Ketosis: A condition characterized by an abnormally elevated concentration of KETONE BODIES in the blood (acetonemia) or urine (acetonuria). It is a sign of DIABETES COMPLICATION, starvation, alcoholism or a mitochondrial metabolic disturbance (e.g., MAPLE SYRUP URINE DISEASE).3-Hydroxybutyric Acid: BUTYRIC ACID substituted in the beta or 3 position. It is one of the ketone bodies produced in the liver.Diabetic Ketoacidosis: A life-threatening complication of diabetes mellitus, primarily of TYPE 1 DIABETES MELLITUS with severe INSULIN deficiency and extreme HYPERGLYCEMIA. It is characterized by KETOSIS; DEHYDRATION; and depressed consciousness leading to COMA.Ketone Bodies: The metabolic substances ACETONE; 3-HYDROXYBUTYRIC ACID; and acetoacetic acid (ACETOACETATES). They are produced in the liver and kidney during FATTY ACIDS oxidation and used as a source of energy by the heart, muscle and brain.Ketogenic Diet: A course of food intake that is high in FATS and low in CARBOHYDRATES. This diet provides sufficient PROTEINS for growth but insufficient amount of carbohydrates for the energy needs of the body. A ketogenic diet generates 80-90% of caloric requirements from fats and the remainder from proteins.Hydroxybutyrates: Salts and esters of hydroxybutyric acid.Acetoacetates: Salts and derivatives of acetoacetic acid.Parturient Paresis: A disease of pregnant and lactating cows and ewes leading to generalized paresis and death. The disease, which is characterized by hypocalcemia, occurs at or shortly after parturition in cows and within weeks before or after parturition in ewes.Acidosis: A pathologic condition of acid accumulation or depletion of base in the body. The two main types are RESPIRATORY ACIDOSIS and metabolic acidosis, due to metabolic acid build up.Cattle Diseases: Diseases of domestic cattle of the genus Bos. It includes diseases of cows, yaks, and zebus.Butylene Glycols: 4-carbon straight chain aliphatic hydrocarbons substituted with two hydroxyl groups. The hydroxyl groups cannot be on the same carbon atom.Peripartum Period: The period shortly before, during, and immediately after giving birth.Acetone: A colorless liquid used as a solvent and an antiseptic. It is one of the ketone bodies produced during ketoacidosis.Diabetic Coma: A state of unconsciousness as a complication of diabetes mellitus. It occurs in cases of extreme HYPERGLYCEMIA or extreme HYPOGLYCEMIA as a complication of INSULIN therapy.Megasphaera: A genus of gram-negative bacteria in the family ACIDAMINOCOCCACEAE, found in the RUMEN of SHEEP and CATTLE, and also in humans.Abomasum: The fourth stomach of ruminating animals. It is also called the "true" stomach. It is an elongated pear-shaped sac lying on the floor of the abdomen, on the right-hand side, and roughly between the seventh and twelfth ribs. It leads to the beginning of the small intestine. (From Black's Veterinary Dictionary, 17th ed)Selenomonas: Curved bacteria, usually crescent-shaped rods, with ends often tapered, occurring singly, in pairs, or short chains. They are non-encapsulated, non-sporing, motile, and ferment glucose. Selenomonas are found mainly in the human buccal cavity, the rumen of herbivores, and the cecum of pigs and several rodents. (From Bergey's Manual of Determinative Bacteriology, 9th ed)Hyperglycemic Hyperosmolar Nonketotic Coma: A serious complication of TYPE 2 DIABETES MELLITUS. It is characterized by extreme HYPERGLYCEMIA; DEHYDRATION; serum hyperosmolarity; and depressed consciousness leading to COMA in the absence of KETOSIS and ACIDOSIS.Placenta, Retained: A placenta that fails to be expelled after BIRTH of the FETUS. A PLACENTA is retained when the UTERUS fails to contract after the delivery of its content, or when the placenta is abnormally attached to the MYOMETRIUM.KetonesLameness, Animal: A departure from the normal gait in animals.Fatty Acids, Nonesterified: FATTY ACIDS found in the plasma that are complexed with SERUM ALBUMIN for transport. These fatty acids are not in glycerol ester form.DairyingCoenzyme A-Transferases: Enzymes which transfer coenzyme A moieties from acyl- or acetyl-CoA to various carboxylic acceptors forming a thiol ester. Enzymes in this group are instrumental in ketone body metabolism and utilization of acetoacetate in mitochondria. EC 2.8.3.Stomach Diseases: Pathological processes involving the STOMACH.

*  Ketosis in dairy cattle | Cattle Network

Ketosis is one metabolic disease, frequently observed in high producing herds. Ketosis usually occurs within a few days to a ... Ketosis is one metabolic disease, frequently observed in high producing herds. Ketosis usually occurs within a few days to a ... Based on various reports, the incidence of clinical ketosis can range from 2 to 15% and subclinical ketosis from 9 to 34%. ... Several studies have described deleterious effects of ketosis on animal health and reproduction. Clinical ketosis is associated ...

*  How do you achieve a state of "ketosis"? |

Low ketosis is normal, but high levels can be serious. High ketosis occurs when a person eats small amounts... ... Ketosis is a physiological state that occurs as the result of raised blood glucose levels. ... Low ketosis is normal, but high levels can be serious. High ketosis occurs when a person eats small amounts of protein and ... The body goes into a state of ketosis when fat must be broken down for energy. It is characterized by high levels of ketones in ...

*  ketosis

Although a Ketogenic diet can help improve epilepsy in your children, some parents may not want to alter their lifestyle in such a profound way. Various foods can be enjoyed that reduce these episodes without having to develop a strict method of eating. Perhaps your child is visiting a location where a Ketogenic meal is […]. ...

*  Instant Ketosis -

This ketone ester is a synthetic prodrug which, after ingestion, your body naturally breaks down into the ketone bodies beta-hydroxybutyrate (BHB) and acetoacetate (AcAc).

*  Citric acid/ ketosis -

Why does citric acid knock some people out of ketosis??? Does it have anything to do with the KREB cycle? ... Ketosis, voluntary ketosis like you have on a ketogenic diet and not diabetes-induced ketoacidosis, relies on the Cori cycle, ... Citric acid/ ketosis Why does citric acid knock some people out of ketosis???. Does it have anything to do with the KREB cycle ... Ketosis, voluntary ketosis like you have on a ketogenic diet and not diabetes-induced ketoacidosis, relies on the Cori cycle, ...

*  Ketosis study yields surprising results | Dairy Herd Management

You'd think treating fresh cows for ketosis would decrease culling risk, but it did the opposite in this study. ... The researchers say that a better understanding of how ketosis develops and what causes it are needed in order to find ways to ... Treating fresh cows for ketosis failed to prevent, and even increased, the negative consequences often associated with this ...

*  Ketosis Sticks - 3 Fat Chicks on a Diet Weight Loss Community

Ketosis Sticks. So I have used these everytime at the center. And I am always in the negative zone. I tell them I'm at trace ( ... How Long Does It Take To Get Into Ketosis? needtolose4me2. Carb Counters. 30. 11-15-2013 06:37 PM. ... They also said that not everyone will go into ketosis and just like Joy, I am losing so I don't worry about it. I think that my ... My center told me that they have you use the sticks to make sure you don't go too deep into ketosis, meaning the really deep ...

*  Low-Carb Foods | Low-Carb Recipes | Ketosis | CarbSmart

Low-Carb Cookbooks for High Fat LCHF Nutritional Ketosis. ... 12 Healthy High-Fat Foods Perfect For Nutritional Ketosis *. ... By Dana Carpender • Leave a Comment • Filed Under: Fat Fast, Fat Fast Recipes, Nutritional Ketosis, Recipes, Salad Recipes • ... 10 Unexpected Health Benefits Of Nutritional Ketosis Besides Weight Loss *. Peach Cobbler Low-Carb Recipe From CarbSmart Grain- ... How to Add More Fat to Your Low Carb Diet for Nutritional Ketosis ...

*  Pains in lower back/ kidney area when in ketosis? |

I know that ketosis does put stress on the kidneys which is why many people disaprove of VLCDs but I really wanna lose teh rest ... Pains in lower back/ kidney area when in ketosis?. Discussion in 'Cambridge Weight Plan' started by DebbieMiller1981, 9 May ...

*  The Clinical Use of Nutritional Ketosis in Treating Cancer And Many Chronic Diseases « Socio-Economics History Blog

Mercola, Story at-a-glance- * Nutritional ketosis, which involves eating a high-quality, high-fat diet ... The Clinical Use of Nutritional Ketosis by Dr. ... Nutritional ketosis, which involves eating a high-quality, high ... The Clinical Use of Nutritional Ketosis in Treating Cancer And Many Chronic Diseases. *The Clinical Use of Nutritional Ketosis ... Clinical Use of Nutritional Ketosis. Part of Drisko's focus has been on the clinical use of nutritional ketosis, which is what ...

*  Patrick Arnold » Exogenous Insulin and Ketosis

By Farmacist Editors Note: The following article is a guest blog by Farmacist. When ketogenic diets came back in vogue in the 90's, bodybuilders were looking for ways to speed up getting into ketosis. The diets were cyclic and it took several unpleasant days to get into full blown ketosis. Glucose disposal agents were typically used, but some of the more daring folks used low doses of insulin to speed the process. The idea was, the faster you get into ketosis, the more fat burning days you would get per cycle. This of course carried the risk of overdosing and becoming hypoglycemic, so this was probably not done by too many people.. Today something like Ketoforce could get your ketones raised quickly, to avoid the unpleasant shift from glucose to ketones for fuel by your brain. That said there are some interesting things that could be, in theory, done with insulin and ketosis.. Disclaimer: Insulin can kill you, do not use insulin without ...

*  Metabolically Challenged: Ketosis

I've talked about Ketosis in a number of posts and I felt that a little bit more clarity of this key metabolic process was necessary. More so, since there are a number of ignorant health care providers, who get scared of the term, and will pass on their ignorance and their fears to us. If you will care to note, everywhere I've mentioned the term, I've referred to it as "Nutritional Ketosis". That because, Nutritional ketosis is very different to "Ketoacidosis" which is what many would confuse this state with - mostly doctors (yes doctors). So let's look at what is so different between them and why Ketosis is such a beneficial state for us to be in ...

*  Side Effects of Ketosis

Normally, our body gets energy from various dietary sources of carbohydrates. When the carbohydrate content is reduced in a diet, our body is forced to obtain energy by burning the body fat. The specialized metabolic mechanisms for the breakdown of fat molecules in the adipose tissue are activated in order to achieve this. Such a breakdown of fats to obtain energy results in the production of beta-hydroxybutyrate (BHB) and acetoacetate. Acetoacetate further breaks down to acetone which is utilized to fulfill the energy requirements of the body. These three molecules are collectively termed as ketone bodies. In the initial few days, the excess ketone bodies are excreted through urine, and the level of ketones in the body remain balanced. Later on, the continuous rise in ketones leads to their accumulation in the body. This condition is referred to as ketosis. The state of ketosis is not only associated with weight loss, it may also prove beneficial in the treatment of ...

*  Measuring ketosis with an Arduino | Hackaday

I use the Ketonix multi-use ketone breath analyzer ($149) which provides color-coded and numerical readings in ppmv. I have not found a clear conversion path but a reading of "50" ppmv is roughly the same as 1.0 mmol/L. That's my minimal goal.. Loss of electrolytes is a natural result of ketosis. Simply add sodium (sea salt) and potassium (lite salt) and magnesium (Calm) should you feel a lack of energy, or irritable.. Nutritional Ketosis is the perfectly normal reaction of the body to lack of glucose, either through famine or carb restriction. Normal body cells switch readily to ketones. Cancer cells do not, and starve (an added benefit). Ketones protect brain cells from oxidation (dementia) and muscle mass from cannibalization. The body burns fat for fuel, beginning with the worst type (hard, brown) and then moving to the less harmful (squishy, white).. NK has nothing to do with ketoacidosis, the dangerous state of high blood glucose AND high ketones, which can occur in ...

*  Is Ketosis Safe Long Term? #ancientcoins - Bella Gurner

Is Ketosis Safe Long Term? If you enjoyed this videovisit the ancient Greek and Roman Coin Collecting youtube channel for more great videos. for more The blog post Is Ketosis Safe Long Term? #ancientcoins is republished from Favorite Video Site

*  Ketosis breath - what is it and how to cure it -

Ketosis breath is something that many people on a low carb diet can experience. Learn what it is, how to avoid it and if there is any possible cure for it.

*  Pearson Report: K is for Ketosis - how it works.

The carbohydrates you eat are converted to glucose, which is the body's primary source of energy. Whenever your intake of carbohydrates is limited to a certain range, for a long enough period of time, you reach a point where your body draws on its alternate energy system, fat stores, for fuel. This means the body burns fat and turns it into a source of fuel called ketones. (Ketones are produced whenever body fat is burned.) ...

*  Homeopathic spray....Should I be in Ketosis???

Hi there Does anyone ever check their ketones while on this diet, I read somewhere that it is a good way of telling if your body is really burning fat and not j

*  Ketosis and Brazilian Jiu Jitsu - The BJJ Caveman

Disclosures: Please note that some of the links provided are affiliate links, and at no additional cost to you, I will earn a commission if you decide to make a purchase. Please understand that I have experience with all of these products. If they're books, I've read them cover to cover, and if they're products or supplements, I've used and/or continue to use them, and I am not shy about giving my honest opinion of them, positive or negative. The small commissions I make help me out a tiny bit, and if you've found my site helpful then feel free to purchase these products through the links I've provided. If not, that's fine too, no pressure, I'll still continue to write! Please do not spend any money on these products unless you feel you need them or that they will help you achieve your goals.. We are a participant in the Amazon Services LLC Associates Program, an affiliate advertising program designed to provide a means for us to earn fees by linking to and affiliated ...

*  A Heart at Peace Gives Life to the Body: Ketosis in a nutshell

Hello. My name is Kari. I graduated from Eastern State Florida College as a Phlebotomist. I am currently working. I attending school to get my AA and then continue my education for occupational therapy. This blog is to write out life's pains and stresses, But also successes and achievements ...

*  Dysnutrition: Fasting and ketosis

Ces transformations résultent de l'industrialisation et de l'utilisation de végétaux et d'animaux non sauvages profondément transformés pour en faire des produits alimentaires. Un grand nombre de ces produits ne sont adaptés ni à notre physiologie ni à notre génomique et produisent des pathologies chroniques même s'ils permettent un apport calorique stable voire excessif pour les pays industrialisés et émergents. Toute la problématique est là. Analyser comment ces transformations bouleversent nos régulations cérébrales et générales et entraînent l'obésité, le diabète, la majorité des cancers, l'athérome et les démences chez certains d'entre nous. ...

KetosisDiabetic ketoacidosisKetogenesisCyclic ketogenic diet: A cyclic ketogenic diet (or carb-cycling) is a low-carbohydrate diet with intermittent periods of high or moderate carbohydrate consumption. This is a form of the general Ketogenic diet that is used as a way to maximize fat loss while maintaining the ability to perform high-intensity exercise.Polyhydroxybutyrate: Polyhydroxybutyrate (PHB) is a polyhydroxyalkanoate (PHA), a polymer belonging to the polyesters class that are of interest as bio-derived and biodegradable plastics.Frieder W.Acetoacetic acidDowner (animal): A downer is an animal, usually livestock, that cannot stand on its own and therefore is to be killed. A downed animal, one that is unable to stand, is not necessarily a downer.AcidosisSecoisolariciresinol diglucosidePeripartum cardiomyopathyPhenylacetoneDiabetic comaAbomasitis: Abomasitis (abomasal bloat) is a relatively rare ruminant disease characterized by inflammation of abomasum in young calves, lambs, and goat kids. It occurs with gastroenteritis but is seldom diagnosed as a separate condition, and its causes are as yet unknown.Hyperosmolar syndrome: Hyperosmolar syndrome or diabetic hyperosmolar syndrome is a medical emergency caused by a very high blood glucose level.Friesian horseDiketoneSidebone: Sidebone is a common condition of horses, characterized by the ossification of the collateral cartilages of the coffin bone. These are found on either side of the foot protruding above the level of the coronary band.Supercow (dairy): Supercow (or super cow) is a term used in the dairy industry to denote lines or individual animals that have superior milk production: that is, which produce more milk per day, or in some cases produce more fat per gallon of milk. Biology of the super cow.Reactive gastropathy: In gastroenterology, reactive gastropathy, also chemical gastropathy, is an abnormality in the stomach caused by chemicals, e.g.

(1/154) Effect of acute ketosis on the endothelial function of type 1 diabetic patients: the role of nitric oxide.

In type 1 diabetic patients, acute loss of metabolic control is associated with increased blood flow, which is believed to favor the development of long-term complications. The mechanisms for inappropriate vasodilation are partially understood, but a role of endothelium-derived nitric oxide (NO) production can be postulated. We assessed, in type 1 diabetic patients, the effect of the acute loss of metabolic control and its restoration on forearm endothelial function in 13 type 1 diabetic patients who were studied under conditions of mild ketosis on two different occasions. In study 1, after basal determination, a rapid amelioration of the metabolic picture was obtained by insulin infusion. In study 2, seven type 1 diabetic patients underwent the same experimental procedure, except that fasting plasma glucose was maintained constant throughout. Basal plasma venous concentrations of nitrites/nitrates (NO2- + NO3-) were determined both before and after intravenous insulin infusion. Endothelium-dependent and -independent vasodilation of the brachial artery was assessed by an intra-arterial infusion of N(G)-monomethyl-L-arginine (L-NMMA) and sodium nitroprusside (SNP), respectively. The same parameters were determined in 13 control subjects at baseline conditions and during a hyperinsulinemic-euglycemic glucose clamp. Baseline forearm blood flow (4.89 +/- 0.86 vs. 3.65 +/- 0.59 ml x (100 ml tissue)(-1) x min(-1)) and NO2- + NO3- concentration (30 +/- 8 vs. 24 +/- 3 micromol/l) were higher in type 1 diabetic patients than in control subjects (P < 0.05). Insulin infusion was associated with lower forearm blood flow and plasma (NO2- + NO3-) concentration (P < 0.05), irrespective of the prevailing glucose levels, as compared with patients under ketotic conditions. The responses to L-NMMA were significantly lower in type 1 diabetic patients during euglycemia and hyperglycemic hyperinsulinemia (-11 +/- 5 and -10 +/- 4%, respectively, of the ratio of the infused arm to the control arm) than in control subjects at baseline (-18 +/- 6%, P < 0.05) and during hyperinsulinemia (-32 +/- 11%, P < 0.01). We conclude that the acute loss of metabolic control is associated with a functional disturbance of the endothelial function characterized by hyperemia and increased NO release during ketosis and blunted NO-mediated vasodilatory response during restoration of metabolic control by intravenous insulin. This functional alteration is unlikely to be explained by hyperglycemia itself.  (+info)

(2/154) Human brain beta-hydroxybutyrate and lactate increase in fasting-induced ketosis.

Ketones are known to constitute an important fraction of fuel for consumption by the brain, with brain ketone content generally thought to be low. However, the recent observation of 1-mmol/L levels of brain beta-hydroxybutyrate (BHB) in children on the ketogenic diet suggests otherwise. The authors report the measurement of brain BHB and lactate in the occipital lobe of healthy adults using high field (4-T) magnetic resonance spectroscopy, measured in the nonfasted state and after 2- and 3-day fasting-induced ketosis. A 9-mL voxel located in the calcarine fissure was studied, detecting the BHB and lactate upfield resonances using a 1H homonuclear editing sequence. Plasma BHB levels also were measured. The mean brain BHB concentration increased from a nonfasted level of 0.05 +/- 0.05 to 0.60 +/- 0.26 mmol/L (after second day of fasting), increasing further to 0.98 +/- 0.16 mmol/L (after the third day of fasting). The mean nonfasted brain lactate was 0.69 +/- 0.17 mmol/L, increasing to 1.47 +/- 0.22 mmol/L after the third day. The plasma and brain BHB levels correlated well (r = 0.86) with a brain-plasma slope of 0.26. These data show that brain BHB rises significantly with 2- and 3-day fasting-induced ketosis. The lactate increase likely results from ketones displacing lactate oxidation without altering glucose phosphorylation and glycolysis.  (+info)

(3/154) Reduction in serum lecithin:cholesterol acyltransferase activity prior to the occurrence of ketosis and milk fever in cows.

Lecithin:cholesterol acyltransferase (LCAT) is the enzyme responsible for production of cholesteryl esters in plasma. The LCAT activity is reduced in cows with fatty liver developed during the nonlactating stage and those with the fatty liver-related postparturient diseases such as ketosis. The purpose of the present study was to examine whether reduced LCAT activity during the nonlactating stage could be detected before the occurrence of postparturient diseases. Sera from 24 cows were collected at approximately 10-day intervals from -48 to +14 days from parturition. Of the 24 cows, 14 were apparently healthy, whereas 7 had ketosis and 3 had milk fever at around parturition. Of the 14 healthy cows, 7 had unaltered LCAT activity during the observation period, whereas 7 showed reduced activity from -20 to +14 days. Ketosis and milk fever occurred at from -3 to +10 days, but reductions of LCAT activity in diseased cows had already been observed from days -20 to 0. These results suggest that LCAT activity is virtually unaffected during the peripartum period at least in some healthy cows and also that the reduction in LCAT activity can be detected before the occurrence of ketosis and milk fever.  (+info)

(4/154) Decreased concentration of serum apolipoprotein C-III in cows with fatty liver, ketosis, left displacement of the abomasum, milk fever and retained placenta.

Apolipoprotein (apo) C-III is a low molecular mass protein mainly distributed in the high-density lipoprotein (HDL) fraction. In cows with postparturient diseases such as ketosis, concentrations of cholesterol, phospholipids and apoA-I and the activity of lecithin:cholesterol acyltransferase, which are mainly distributed in or functionally associated with HDL, are reduced. The purpose of the present study was to examine whether the serum concentration of apoC-III was similarly decreased in the postparturient diseases. Compared with healthy controls, the apoC-III concentration was significantly (P<0.01) decreased in cows with fatty liver, ketosis, left displacement of the abomasum, milk fever and retained placenta. Concentrations of apoC-III in the HDL fractions from diseased cows were also lower than in controls. Of the diseased cows, the decreased apoC-III concentration was particularly distinct in cows with milk fever. Increased nonesterified fatty acid and reduced free cholesterol, cholesteryl ester and phospholipid concentrations were observed in cows with milk fever, as in the other diseased cows. The decrease in the apoC-III concentration is suggested to be closely associated with the postparturient disorders, in particular with milk fever.  (+info)

(5/154) Diabetic ketoacidosis associated with recurrent pulmonary edema and rhabdomyolysis in a patient with Turner's syndrome.

Turner's syndrome is a condition involving total or partial absence of one X chromosome and has been associated with a number of diseases including non insulin dependent diabetes mellitus, abnormalities of glucose metabolism and hypothreosis. There have been many case reports in which Turner's syndrome is associated with type 2 diabetes, but the association with type 1 diabetes and/or life threatening complications is very rare. We present an unusual case of a patient with Turner's syndrome who has type 1 diabetes and is complicated with ketoacidosis, severe acute and recurrent pulmonary edema and rhabdomyolysis.  (+info)

(6/154) Field study of dairy cows with reduced appetite in early lactation: clinical examinations, blood and rumen fluid analyses.

The study included 125 cows with reduced appetite and with clinical signs interpreted by the owner as indicating bovine ketosis 6 to 75 days postpartum. Almost all of the cows were given concentrates 2 to 3 times daily. With a practitioners view to treatment and prophylaxis the cows were divided into 5 diagnostic groups on the basis of thorough clinical examination, milk ketotest, decreased protozoal activity and concentrations, increased methylene blue reduction time, and increased liver parameters: ketosis (n = 32), indigestion (n = 26), combined ketosis and indigestion (n = 29), liver disease combined with ketosis, indigestion, or both (n = 15), and no specific diagnosis (n = 17). Three cows with traumatic reticuloperitonitis and 3 with abomasal displacement were not grouped. Nonparametric methods were used when groups were compared. Aspartate aminotransferase, glutamate dehydrogenase, gamma-glutamyl transferase and total bilirubin were elevated in the group with liver disease. Free fatty acids were significantly elevated in cows with ketosis, compared with cows with indigestion. Activity and concentrations of large and small protozoas were reduced, and methylene blue reduction time was increased in cows with indigestion. The rumen fluid pH was the same for groups of cows with and without indigestion. Prolonged reduced appetite before examination could have led to misclassification. Without careful interpretation of the milk ketotest, many cases with additional diagnoses would have been reported as primary ketosis. Thorough clinical examination together with feasible rumen fluid examination and economically reasonable blood biochemistry did not uncover the reason(s) for reduced appetite in 14% of the cows. More powerful diagnostic methods are needed.  (+info)

(7/154) Lipid metabolism in the cow during starvation-induced ketosis.

1. Concentrations and compositions of liver, serum and milk lipids of cows were measured during 6 days' starvation and serum lipids during 60 days' re-feeding. 2. The concentration of free fatty acid in serum increased fivefold during starvation. 3. The content of total lipid in liver (g/100g of liver dry matter) doubled owing to a 20-fold increase in triglyceride, an eightfold increase in cholesterol ester, a three fold increase in free fatty acid and a 20% increase in cholesterol. There were no changes in the content or composition of liver phospholipids. 4. Starvation lowered the concentrations of total lipid, phospholipid and cholesterol ester of dextran sulphate-precipitable serum lipoproteins. Total lipid and cholesterol ester concentrations in lipoproteins of d greater than 1.055 and in lipoproteins not precipitable by dextran sulphate decreased from day 4 of the starvation period and during the first 20 days' re-feeding. 5. During starvation there were decreases in percentages of stearic acid and increases in oleic acid in serum free fatty acids and triglycerides and in liver neutral lipid. 6. Throughout starvation total milk lipid yield decreased, yields and percentages of C4-14 fatty acids decreased and percentages of C18 fatty acids increased. 7. It is suggested that accumulation of triglyceride in liver may be caused by increased uptake of plasma free fatty acids without corresponding increase in lipoprotein secretion.  (+info)

(8/154) Breath acetone as a measure of systemic ketosis assessed in a rat model of the ketogenic diet.

BACKGROUND: The mechanism of a high-fat, low-carbohydrate ketogenic diet (KD) in alleviating drug-resistant epilepsy is unknown but may be related to systemic ketosis induced under this treatment. The need for frequent measurement of systemic ketosis, which is essential for improving maintenance of the KD in patients and for studying mechanism of the KD action, has prompted us to validate the breath acetone test as a fast, reliable, and noninvasive tool for ketosis assessment. METHODS: A rat model of the KD that allowed frequent blood sampling was used to investigate how well breath acetone reflects plasma beta-hydroxybutyrate (beta-HBA), the most commonly measured ketone body. Rat pups (20 days of age) were introduced to and maintained on a KD or control diet for 33 days. During this period, breath acetone, plasma beta-HBA, blood glucose, and body weight were measured approximately every 4th day. A correlational analysis of breath acetone and plasma beta-HBA was conducted. RESULTS: Breath acetone was found to be a significant predictor of plasma beta-HBA over a clinically relevant range of beta-HBA concentrations (r(2) = 0.75; P <0.001). We have proposed a general formula that allows the value of plasma beta-HBA to be estimated based on breath acetone measurement. CONCLUSIONS: Breath acetone is an accurate measure of mild to moderate systemic ketosis. The noninvasive nature of this test will be useful for day-to-day implementation of the KD, searching for better forms of this diet, and understanding the role of ketosis in the mechanism of the KD action.  (+info)

state of ketosis

  • The body goes into a state of ketosis when fat must be broken down for energy. (
  • Initiating a state of ketosis can cause fat loss, but it also can be medically serious and cause a person to have acidic blood. (
  • The state of ketosis is not only associated with weight loss, it may also prove beneficial in the treatment of Alzheimer's disease, Parkinson's disease, and epilepsy seizures in children. (
  • This process happens when you enter the state of ketosis and this is what causes the bad ketosis breath smell. (

nutritional ketosis

  • Part of Drisko's focus has been on the clinical use of nutritional ketosis , which is what I recommend for nearly everyone to improve their health. (
  • This is my 10th day straight of being in nutritional ketosis it looks like my guess from last night was pretty on the mark. (
  • to determine if I am in the range of nutritional ketosis (0.5 mmol- 3.0 mmol). (

ketogenic diet

  • Ketosis, voluntary ketosis like you have on a ketogenic diet and not diabetes-induced ketoacidosis, relies on the Cori cycle, and lets the Krebs cycle completely collapse. (


  • I find the ketosis very interesting because I have epilepsy. (


  • This process of burning a lot of fat to produce these ketones is known as ketosis. (


  • Bad breath is another side effect of ketosis which is triggered by the abnormal increase in the ketone bodies. (
  • In fact, breath acetone serves as a diagnostic measure for ketosis. (
  • People who eat according to keto diet sometimes get something called ketosis breath. (
  • The ketosis breath smell can be very special and could affect your social life. (
  • It could also be something that irritates you personally if you do not like the ketosis breath taste or smell. (
  • The reason people get a bad ketosis breath smell is usually caused by that when your body is burning fat certain chemicals are released in your breath. (
  • It is however not only a bad thing that you have a bad ketosis breath smell. (
  • However it is easy for people that notice you have a bad ketosis breath to assume you have poor oral hygiene. (
  • You should know that no matter how much you brush your teeth, floss or scrape your tongue this will not help to reduce the ketosis breath smell. (
  • There is no perfect ketosis breath cure. (
  • The best way to reduce the bad ketosis breath symptoms is to use mouthwashes or chewing gum. (
  • This is however not a 100% solution and it is still possible that people will notice your ketosis breath. (
  • You could also try to drink more water and swish it around in your mouth to clean out some particles that could cause the ketosis breath smell. (
  • If you still are not able to get rid of the bad ketosis breath the final step could be to increase your intake of carbohydrates in your diet. (
  • It is not the best solution for someone on a keto diet but it might be the only way for you to finally get rid of that ketosis breath smell. (


  • They were at the highest risk of developing clinical and subclinical ketosis compared to cows classified as either a moderate or thin BCS prior to calving. (
  • Treating fresh cows for ketosis failed to prevent, and even increased, the negative consequences often associated with this disease, according to research from the University of Minnesota. (
  • The researchers say that a better understanding of how ketosis develops and what causes it are needed in order to find ways to manage its consequences on cows. (


  • I think some early Atkins dieters were drinking lots of diet soda and couldn't get into ketosis. (
  • Atkins concluded that caffeine was the problem when the real culprit was something else in the soda (again, citric acid and nutrasweet seem to keep some people out of ketosis). (
  • I did Atkins many years ago and I NEVER got into ketosis. (


  • Lose weight fast with my pro tips and an easy low carb menu plan for getting into ketosis in three days or less. (


  • Ketosis usually occurs within a few days to a few weeks after calving. (
  • Ketosis is a physiological state that occurs as the result of raised blood glucose levels. (
  • High ketosis occurs when a person eats small amounts of protein and carbohydrates while eating large amounts of fat. (


  • Clinical ketosis is associated with an increase of 2 to 3 days to first service and a 4 to 10% reduction in pregnancies per AI at first service. (


  • I stick to the program to a T (except for like 2 dinners where I had a few bites of something off program), and I'm just wondering why my body isn't going into ketosis. (
  • The trick to put our body into ketosis mode is to avoid all sources of carbohydrates from rice, grains, sweets, potatoes and all those starchy food. (
  • More fat consumption will make sure you eat less and thereby reduces your protein and carbohydrate intake thus putting your body into optimal ketosis. (
  • Ketosis is a process that the body does on an everyday basis, regardless of the number of carbs you eat. (


  • Any factor resulting in a reduction of dry matter intake (DMI) increases the risk for ketosis. (


  • Many people in ketosis drink alot of diet soda, which contains citric acid, and these people (including myself) haven't been kicked out of ketosis by it. (
  • You can use this as an indication that you have actually come into ketosis which is considered as a positive thing when eating according to a keto diet. (


  • It is characterized by low blood glucose, excess ketone bodies in blood and urine, lack of appetite, either lethargy or excitability, weight loss, depressed milk production and occasionally, in cases of severe ketosis, incoordination and neurologic signs. (
  • What can I do to go into ketosis and make my weight loss increase? (
  • I know that ketosis does put stress on the kidneys which is why many people disaprove of VLCDs but I really wanna lose teh rest of the weight so don't want to stop! (
  • The best thing is to go to your physician and ask him for advice and also try to get a complete check-up done before starting ketosis for weight loss. (
  • How Does Ketosis Help in Weight Loss? (
  • Whenever I am on hcg, I rarely go into 'trace' ketosis, even tho I am loosing weight. (


  • Why does citric acid knock some people out of ketosis? (
  • many people NEVER go into ketosis -- and the center I went to never even used the sticks. (


  • Other researchers have identified an association between ketosis and an increased incidence of ovarian cysts. (


  • You should drink lots of water as ketosis often causes dehydration, so you need to drink more water than you ever did. (


  • Another best ketosis friendly food is fish like sardines that have fewer carbohydrates and pack all the essential nutrients . (


  • Ketosis is one metabolic disease, frequently observed in high producing herds. (
  • Low ketosis is normal, but high levels can be serious. (


  • Ketosis actually helps some patients with seizure control. (


  • Several studies have described deleterious effects of ketosis on animal health and reproduction. (


  • They also said that not everyone will go into ketosis and just like Joy, I am losing so I don't worry about it. (
  • I just wanted to let you know that, although everyone is different, I stay in ketosis everyday. (


  • Ketosis has also been associated with heart palpitations. (


  • However, I just haven't seen any evidence that just the presence of Citric Acid could interfere with ketosis, especially as the main source of oxaloacetate is Pyruvate. (



  • Managing BCS towards the end of the previous lactation is an important management practice to minimize ketosis and other postpartum metabolic diseases. (


  • My center told me that they have you use the sticks to make sure you don't go too deep into ketosis, meaning the really deep pink to purple. (