Herpes Zoster Oticus
Herpes Zoster
Herpes Zoster Ophthalmicus
Herpes Zoster Vaccine
Neuralgia, Postherpetic
Herpesvirus 3, Human
Chickenpox
Herpes Simplex
Chickenpox Vaccine
Acyclovir
Labyrinth Diseases
Vertigo
Cochlea
Ear, Inner
Ramsay Hunt syndrome associated with brain stem enhancement. (1/13)
Postcontrast T1-weighted MR images in a patient with Ramsay Hunt syndrome showed an enhancing lesion in the region of the nucleus of the pontine facial nerve and abnormal enhancement of the intrameatal, labyrinthine, and tympanic facial nerve segments and of the geniculate ganglion, as well as enhancement of the vestibulocochlear nerve and parts of the membranous labyrinth. This enhancement most probably resulted from a primary neuritis of the intrameatal nerve trunks of the seventh and eighth cranial nerves. (+info)Sympathetic contralateral vestibulopathy after unilateral zoster oticus. (2/13)
A unique case of initially right sided varicella zoster induced Ramsay-Hunt syndrome with complete vestibular loss is reported. The patient subsequently developed deficits of the left vestibule 5 months later. An autoimmune pathogenesis of the left vestibular failure rather than bilateral varicella zoster infection was suggested by the following data: (1) no evidence of vesicular eruptions on the left auricle and the virtual absence of antiviral antibodies after onset of bilateral vestibulopathy; (2) prompt response of the left vestibule to immunosuppressive therapy with corticosteroids; and (3) presence of atypical nervous tissue specific autoantibodies against a 45 kDa protein. (+info)Quantitation of varicella-zoster virus DNA in patients with Ramsay Hunt syndrome and zoster sine herpete. (3/13)
Varicella-zoster virus (VZV) reactivation causes facial nerve palsy in Ramsay Hunt syndrome (RHS) and zoster sine herpete (ZSH) with and without zoster rash, respectively. In the present study, we analyzed the VZV DNA copy number in saliva samples from 25 patients with RHS and 31 patients with ZSH using a TaqMan PCR assay to determine differences in the viral load between the two diseases. VZV copy number in saliva peaked near the day of the appearance of zoster in patients with RHS. Consequently, VZV DNA was less frequently detected in patients with RHS who exhibited facial palsy several days after the appearance of zoster. These findings suggest that the VZV load in saliva samples reflects the kinetics of viral reactivation in patients with RHS. In addition, VZV DNA was equally detected in saliva from patients with RHS and ZSH, and there was no significant difference in the highest viral copy number between patients with RHS and those with ZSH. The VZV load does not appear to reflect a major difference between RHS and ZSH. (+info)A case of Ramsay Hunt-like syndrome caused by herpes simplex virus type 2. (4/13)
We report an immunocompetent patient with recurrent auricular and facial vesicles associated with painful paresthesias and facial paralysis, consistent with Ramsay Hunt syndrome, due to herpes simplex virus (HSV) type 2. Clinical and laboratory-proven acyclovir resistance developed during therapy. Immunologic assays revealed normal reactivity to HSV-2. (+info)Unilateral facial swelling caused by Ramsay Hunt syndrome resembles odontogenic infection. (5/13)
Facial cellulitis and swellings of the head and neck are worrisome signs of odontogenic infection, which can be life threatening. Most head and neck infections are caused by bacterial pathogens. When treating such infections, dentists must also be aware of possible viral or fungal causes and their associated presentations. This report documents a case of viral infection that initially resembled a bacterial odontogenic infection. It is intended to familiarize dentists with the Ramsay Hunt syndrome and the need for prompt recognition and early definitive treatment. (+info)Prompt contrast enhancement of cerebrospinal fluid space in the fundus of the internal auditory canal: observations in patients with meningeal diseases on 3D-FLAIR images at 3 Tesla. (6/13)
We speculated that meningeal pathologies might facilitate the permeability of cranial nerves at the fundus of the internal auditory canal (IAC), causing prompt enhancement after administration of Gd-DTPA. Using a 3D- fluid-attenuated inversion recovery (FLAIR) sequence, we evaluated the enhancement of the cerebrospinal fluid (CSF) space in the IAC fundus 10 min after Gd-DTPA administration in patients with meningeal diseases. Twenty patients (aged 22 to 79 years) were divided into 2 groups, a group with meningeal disease comprising 9 patients with meningeal abnormalities (6, tumor dissemination; 3, infection) and a control group of 11 patients with unilateral IAC pathology whose healthy sides were included as controls. Six of the 9 patients in the group with meningeal disease showed bilateral enhancement; one showed unilateral enhancement. None of the control group showed enhancement in the healthy side. One patient with Ramsay-Hunt syndrome showed only ipsilateral enhancement. Enhancement in the IAC fundus was frequently observed in patients with meningeal disease, even just 10 min after administration of contrast agent. This enhancement in the IAC fundus was never visible on T1-weighted 3D-FLASH images. (+info)Correlation between MRI and operative findings in Bell's palsy and Ramsay Hunt syndrome. (7/13)
PURPOSE: To investigate the correlation between gadolinium enhanced magnetic resonance image (MRI) results and surgical findings of facial nerves in Bell's palsy and Ramsay Hunt syndrome. MATERIALS AND METHODS: From 1995 to 2004, MRI was performed on 13 patients with Bell's palsy or Ramsay Hunt syndrome, who were offered with surgical decompression of the facial nerve through the middle cranial fossa approach. Gadolinium enhanced MRI was performed on all patients and the enhancement of the facial nerve was evaluated by radiology specialists. Operative findings including the degree of the facial nerve segment swelling were examined. Furthermore, the time interval from the onset of palsy to surgery was evaluated. RESULTS: Swelling of facial nerve segments was found in patients with enhanced facial nerves from MRI. The swelling of the facial nerve in the labyrinthine segment in particular was identified in all patients with enhanced labyrinthine segments in MRI. The intraoperative swelling of geniculate ganglion of facial nerve was found in 78% of patients with enhanced facial segment in MRI (p=0.01). The intraoperative swelling of tympanic segment was observed from fourth to ninth weeks after the onset of palsy. CONCLUSION: MRI enhancement of facial nerves in Bell's palsy and Ramsay Hunt syndrome is associated with the extent of intratemporal lesions of facial nerves, especially in the labyrinthine segment. (+info)Ramsay Hunt syndrome presenting as simple otitis externa. (8/13)
Ramsay Hunt syndrome is a rare complication of herpes zoster in which reactivation of latent varicella zoster virus infection occurs in the geniculate ganglion, causing otalgia, auricular vesicles and peripheral facial paralysis. Because these symptoms do not always present at the onset, this syndrome can be misdiagnosed. We report the case of a patient who was diagnosed with simple otitis externa after presenting to the emergency department (ED) with a 3-day history of right-sided otalgia. Her condition subsequently evolved to include right-sided auricular vesicles and right-sided facial weakness. She presented to the ED again after 2 days and was correctly diagnosed with Ramsay Hunt syndrome. We describe the clinical presentation, diagnostic findings and management of this uncommon but important entity. (+info)Herpes zoster oticus, also known as Ramsay Hunt syndrome type 2, is a viral infection that affects the facial nerve (cranial nerve VII). It is caused by the reactivation of the varicella-zoster virus, which is the same virus responsible for chickenpox. After an initial chickenpox infection, the virus can remain dormant in the body and later reactivate, causing herpes zoster oticus.
In this condition, the virus affects the geniculate ganglion of the facial nerve, leading to inflammation and damage to the nerve fibers. This results in various symptoms, including:
1. Painful rash around the ear, on the face, or in the mouth
2. Facial weakness or paralysis on one side of the face
3. Hearing loss, tinnitus (ringing in the ears), or vertigo (dizziness)
4. Loss of taste sensation on the anterior two-thirds of the tongue
5. Difficulty closing one eye, leading to dryness and irritation
Immediate medical attention is necessary for proper diagnosis and treatment, which typically involves antiviral medications and corticosteroids to reduce inflammation and speed up recovery. Early treatment can help minimize the risk of complications, such as permanent facial nerve damage or hearing loss.
Herpes zoster, also known as shingles, is a viral infection that causes a painful rash. It's caused by the varicella-zoster virus, which also causes chickenpox. After you recover from chickenpox, the virus lies dormant in your nerve cells and can reactivate later in life as herpes zoster.
The hallmark symptom of herpes zoster is a unilateral, vesicular rash that occurs in a dermatomal distribution, which means it follows the path of a specific nerve. The rash usually affects one side of the body and can wrap around either the left or right side of your torso.
Before the rash appears, you may experience symptoms such as pain, tingling, or itching in the area where the rash will develop. Other possible symptoms include fever, headache, fatigue, and muscle weakness. The rash typically scabs over and heals within two to four weeks, but some people may continue to experience pain in the affected area for months or even years after the rash has healed. This is known as postherpetic neuralgia (PHN).
Herpes zoster is most common in older adults and people with weakened immune systems, although anyone who has had chickenpox can develop the condition. It's important to seek medical attention if you suspect you have herpes zoster, as early treatment with antiviral medications can help reduce the severity and duration of the rash and lower your risk of developing complications such as PHN.
Herpes Zoster Ophthalmicus (HZO) is a type of herpes zoster (shingles) infection that affects the ophthalmic division (V1) of the trigeminal nerve. It is caused by the varicella-zoster virus, which also causes chickenpox. After a person recovers from chickenpox, the virus remains inactive in the body and can reactivate later as shingles, often many years after the initial infection.
When the virus reactivates and affects the ophthalmic division of the trigeminal nerve, it can cause a painful rash on the forehead, nose, and around one eye. The rash may be accompanied by other symptoms such as headache, fever, and fatigue. In some cases, HZO can also affect the eye itself, causing inflammation, corneal ulcers, and vision loss if left untreated.
It is important to seek medical attention promptly if you suspect you have HZO, as early treatment with antiviral medications can help reduce the severity of symptoms and prevent complications.
The Herpes Zoster vaccine, also known as the shingles vaccine, is a preventive measure against the reactivation of the varicella-zoster virus (VZV) in individuals who have previously had chickenpox. The vaccine contains a live but weakened form of VZV that boosts the immune system's ability to recognize and fight off the virus, thereby reducing the risk of developing shingles and its complications. It is typically administered as a single dose for people aged 50 and older, or as a two-dose series for those aged 19 and older who have weakened immune systems.
Postherpetic neuralgia (PHN) is a type of neuralgia, which is defined as pain in the distribution of a nerve or nerves. Specifically, PHN is a neuropathic pain condition that develops after an individual has had herpes zoster, also known as shingles. Shingles is caused by the reactivation of the varicella-zoster virus, which lies dormant in the nervous system following chickenpox infection.
PHN is characterized by persistent burning pain, often accompanied by sensory abnormalities such as numbness, tingling, or itching, in the area of the body where shingles occurred. The pain can be severe and debilitating, significantly impacting a person's quality of life. PHN primarily affects older adults and individuals with weakened immune systems.
The exact cause of PHN is not fully understood, but it is believed to result from damage to the affected nerves and their surrounding tissues during the shingles infection. This damage can lead to altered nerve function and increased sensitivity to stimuli, resulting in chronic pain. Treatment for PHN typically involves a combination of medications, such as antidepressants, anticonvulsants, or opioids, as well as topical treatments, physical therapy, and lifestyle modifications to help manage the pain and improve quality of life.
Also known as Varicella-zoster virus (VZV), Herpesvirus 3, Human is a species-specific alphaherpesvirus that causes two distinct diseases: chickenpox (varicella) during primary infection and herpes zoster (shingles) upon reactivation of latent infection.
Chickenpox is typically a self-limiting disease characterized by a generalized, pruritic vesicular rash, fever, and malaise. After resolution of the primary infection, VZV remains latent in the sensory ganglia and can reactivate later in life to cause herpes zoster, which is characterized by a unilateral, dermatomal vesicular rash and pain.
Herpesvirus 3, Human is highly contagious and spreads through respiratory droplets or direct contact with the chickenpox rash. Vaccination is available to prevent primary infection and reduce the risk of complications associated with chickenpox and herpes zoster.
Chickenpox is a highly contagious viral infection caused by the varicella-zoster virus. It is characterized by an itchy, blister-like rash that typically covers the body and can also affect the mouth, eyes, and scalp. The rash progresses through various stages, from red bumps to fluid-filled blisters to scabs, before ultimately healing.
Chickenpox is usually a mild disease in children but can be more severe in adults, pregnant women, and individuals with weakened immune systems. Common symptoms include fever, fatigue, headache, and loss of appetite, which often precede the onset of the rash. The infection typically lasts about 1-2 weeks, and once a person has had chickenpox, they usually develop immunity to future infections.
A vaccine is available to prevent chickenpox, and it is routinely administered to children as part of their childhood vaccination schedule. In some cases, the vaccine may be recommended for adults who have not had chickenpox or been vaccinated previously.
Herpes Simplex is a viral infection caused by the Herpes Simplex Virus (HSV). There are two types of HSV: HSV-1 and HSV-2. Both types can cause sores or blisters on the skin or mucous membranes, but HSV-1 is typically associated with oral herpes (cold sores) and HSV-2 is usually linked to genital herpes. However, either type can infect any area of the body. The virus remains in the body for life and can reactivate periodically, causing recurrent outbreaks of lesions or blisters. It is transmitted through direct contact with infected skin or mucous membranes, such as during kissing or sexual activity.
The chickenpox vaccine, also known as varicella vaccine, is a preventive measure against the highly contagious viral infection caused by the varicella-zoster virus. The vaccine contains a live but weakened form of the virus, which stimulates the immune system to produce a response without causing the disease itself.
The chickenpox vaccine is typically given in two doses, with the first dose administered between 12 and 15 months of age and the second dose between 4 and 6 years of age. In some cases, the vaccine may be given to older children, adolescents, or adults who have not previously been vaccinated or who have never had chickenpox.
The chickenpox vaccine is highly effective at preventing severe cases of the disease and reducing the risk of complications such as bacterial infections, pneumonia, and encephalitis. It is also effective at preventing transmission of the virus to others.
Like any vaccine, the chickenpox vaccine can cause mild side effects such as soreness at the injection site, fever, or a mild rash. However, these side effects are generally mild and short-lived. Serious side effects are rare but may include allergic reactions or severe immune responses.
Overall, the chickenpox vaccine is a safe and effective way to prevent this common childhood disease and its potential complications.
Acyclovir is an antiviral medication used for the treatment of infections caused by herpes simplex viruses (HSV) including genital herpes, cold sores, and shingles (varicella-zoster virus). It works by interfering with the replication of the virus's DNA, thereby preventing the virus from multiplying further. Acyclovir is available in various forms such as oral tablets, capsules, creams, and intravenous solutions.
The medical definition of 'Acyclovir' is:
Acyclovir (brand name Zovirax) is a synthetic nucleoside analogue that functions as an antiviral agent, specifically against herpes simplex viruses (HSV) types 1 and 2, varicella-zoster virus (VZV), and Epstein-Barr virus (EBV). Acyclovir is converted to its active form, acyclovir triphosphate, by viral thymidine kinase. This activated form then inhibits viral DNA polymerase, preventing further replication of the virus's DNA.
Acyclovir has a relatively low toxicity profile and is generally well-tolerated, although side effects such as nausea, vomiting, diarrhea, and headache can occur. In rare cases, more serious side effects such as kidney damage, seizures, or neurological problems may occur. It is important to take acyclovir exactly as directed by a healthcare provider and to report any unusual symptoms promptly.
Labyrinthitis is a medical condition characterized by inflammation of the labyrinth, which is the inner ear's balance- and hearing-sensitive system. It is often caused by an infection, such as a viral or bacterial infection, that spreads to the inner ear. The inflammation can affect the delicate structures of the labyrinth, leading to symptoms such as vertigo (a spinning sensation), dizziness, imbalance, hearing loss, and tinnitus (ringing in the ears). Labyrinthitis can be a serious condition that requires medical attention and treatment.
Labyrinth diseases refer to conditions that affect the inner ear's labyrinth, which is the complex system of fluid-filled channels and sacs responsible for maintaining balance and hearing. These diseases can cause symptoms such as vertigo (a spinning sensation), dizziness, nausea, hearing loss, and tinnitus (ringing in the ears). Examples of labyrinth diseases include Meniere's disease, labyrinthitis, vestibular neuronitis, and benign paroxysmal positional vertigo. Treatment for these conditions varies depending on the specific diagnosis but may include medications, physical therapy, or surgery.
Vertigo is a specific type of dizziness characterized by the sensation that you or your surroundings are spinning or moving, even when you're perfectly still. It's often caused by issues with the inner ear or the balance-sensing systems of the body. Vertigo can be brought on by various conditions, such as benign paroxysmal positional vertigo (BPPV), labyrinthitis, vestibular neuritis, Meniere's disease, and migraines. In some cases, vertigo may also result from head or neck injuries, brain disorders like stroke or tumors, or certain medications. Treatment for vertigo depends on the underlying cause and can include specific exercises, medication, or surgery in severe cases.
The cochlea is a part of the inner ear that is responsible for hearing. It is a spiral-shaped structure that looks like a snail shell and is filled with fluid. The cochlea contains hair cells, which are specialized sensory cells that convert sound vibrations into electrical signals that are sent to the brain.
The cochlea has three main parts: the vestibular canal, the tympanic canal, and the cochlear duct. Sound waves enter the inner ear and cause the fluid in the cochlea to move, which in turn causes the hair cells to bend. This bending motion stimulates the hair cells to generate electrical signals that are sent to the brain via the auditory nerve.
The brain then interprets these signals as sound, allowing us to hear and understand speech, music, and other sounds in our environment. Damage to the hair cells or other structures in the cochlea can lead to hearing loss or deafness.
The inner ear is the innermost part of the ear that contains the sensory organs for hearing and balance. It consists of a complex system of fluid-filled tubes and sacs called the vestibular system, which is responsible for maintaining balance and spatial orientation, and the cochlea, a spiral-shaped organ that converts sound vibrations into electrical signals that are sent to the brain.
The inner ear is located deep within the temporal bone of the skull and is protected by a bony labyrinth. The vestibular system includes the semicircular canals, which detect rotational movements of the head, and the otolith organs (the saccule and utricle), which detect linear acceleration and gravity.
Damage to the inner ear can result in hearing loss, tinnitus (ringing in the ears), vertigo (a spinning sensation), and balance problems.
Ramsay Hunt syndrome type 3
Ramsay Hunt syndrome type 2
Herpes zoster ophthalmicus
Ramsay Hunt syndrome
Geniculate ganglionitis
James Ramsay Hunt
Trigeminal neuralgia
List of neurological conditions and disorders
List of MeSH codes (C10)
Sensorineural hearing loss
List of MeSH codes (C02)
List of diseases (H)
Causes of hearing loss
Shingles
List of skin conditions
Herpes Zoster Oticus: Overview, Pathophysiology, Clinical Manifestations
Herpes Zoster Oticus - Ear, Nose, and Throat Disorders - MSD Manual Consumer Version
Facts about HERPES ZOSTER OTICUS Archives - Dr. Sharad ENT
Vestibular evoked myogenic potentials in herpes zoster oticus | ASN
Herpes Zoster Oticus with Cranial Polyneuropathy without Involvement of Facial Nerve.
Upstate Patient Care | SUNY Upstate Medical University
Shingles | Herpes Zoster | MedlinePlus
Ramsay Hunt syndrome type 3 - Wikipedia
Herpes Zoster Oticus: Overview of Herpes Zoster Oticus, Pathophysiology of Herpes Zoster Oticus, Clinical Manifestations of...
Varicella-Zoster Virus (VZV) Clinical Presentation: History, Physical, Causes
Can Ramsay Hunt Syndrome Cause Eye Damage?
WikiGenes - Decompression, Surgical
Meniere Disease - Ear, Nose, and Throat Disorders - Merck Manuals Consumer Version
urofacial syndrome - Ontology Browser - Rat Genome Database
Labyrinthitis: Background, Etiology, Epidemiology
Dizziness & Vertigo: Causes & Treatments - Life Extension
Ear pain in children - wikidoc
Justin Bieber's remaining 'Justice' world tour dates postponed until at least 2023 amid illness - WPXI
STUDY OF VESTIBULOCOCHLEAR NERVE AFFECTION IN HERPES ZOSTER | 81160
Vol. 3 No. 3 (2016): July-September 2016 | International Journal of Advances in Medicine
Journal of Otolaryngology | Open Access Journals
Specific PHGKB|Rare Diseases PHGKB|PHGKB
DeCS
Don't Lose Your Balance! Your FMEP Dizziness SAMP - Family Medicine Exam Prep
Neurology A-Z List - H on MedicineNet.com
Herpes zoster - WikiLectures
Herpes zoster ophthalmicus - WikEM
Herpes Zoster Soft Palate | Keyword Rankings
Li-Kai Huang - 研究成果 - 臺北醫學大學
Cogan syndrome - wikidoc
Shingles16
- Herpes zoster virus, also known as shingles, results from the reactivation of latent varicella-zoster virus, which infiltrates the sensory ganglia during varicella. (medscape.com)
- Reactivation of latent VZV can result in localized vesicular rash, known as herpes zoster or shingles. (medscape.com)
- Shingles Shingles is a painful skin rash caused by a viral infection that results from reactivation of the varicella-zoster virus, the virus that causes chickenpox. (msdmanuals.com)
- Shingles (herpes zoster) is an infection that causes a painful rash. (medlineplus.gov)
- Varicella-zoster virus stays in your body after you have chickenpox, and it can reactivate years later as shingles. (allaboutvision.com)
- According to the Mayo Clinic , Ramsay Hunt syndrome, also known as herpes zoster oticus, occurs when a shingles outbreak affects the facial nerve close to the ear. (wpxi.com)
- Shingles, or herpes zoster, is an infection caused by the varicella-zoster virus. (drjack.world)
- What is the pathogenesis of herpes zoster (shingles)? (drjack.world)
- It is called the varicella-zoster virus, and it occurs when a shingles outbreak affects the facial nerve located near the ear. (webnewsobserver.com)
- Sibling connection between chickenpox, Ramsay hunt, and shingles is the agent causing the infection that is Herpes zoster virus or varicella-zoster virus. (allthingsmedicine.com)
- There is a close association between shingles and RHS that the causative agent is Varicella zoster. (allthingsmedicine.com)
- Ramsay Hunt syndrome or herpes zoster oticus happens when a shingles outbreak affects your facial nerve near one of the ears. (askapollo.com)
- Herpes Zoster Oticus (Shingles) can infect the ear canal, but is quite rare. (ensg.co.nz)
- With the above chicken pox virus, when the reactivation of the virus happens later it will produce the familiar shingles (herpes zoster) condition. (your-neurologist.com)
- Shingles, also known as herpes zoster, will affect about 1 in 3 Americans during their lifetime. (shinglestalk.com)
- Zostavax, the herpes zoster vaccine , has been shown to significantly decrease the risk of shingles. (shinglestalk.com)
Ophthalmicus2
- Rarely, in cases of herpes ophthalmicus, the virus migrates along the intracranial branches of the trigeminal nerve, causing thrombotic cerebrovasculopathy with severe headache and hemiplegia. (medscape.com)
- Especially, herpes zoster ophthalmicus occurs when the ophthalmic branch of the trigeminal nerve is involved, leading to ocular compli- cations or permanent blindness 5 . (e-jmi.org)
Reactivation of the varicella-zoste5
- Reactivation of the varicella-zoster virus (VZV) along the distribution of the sensory nerves innervating the ear, which usually includes the geniculate ganglion, is responsible for herpes zoster (HZ) oticus. (medscape.com)
- Ramsay Hunt syndrome is a neurological condition caused by the reactivation of the varicella-zoster virus. (allaboutvision.com)
- In 1907, James Ramsay Hunt first postulated that the etiology of herpes zoster oticus was the reactivation of the varicella zoster virus (VZV) in the geniculate ganglion [ 1 ]. (juniperpublishers.com)
- However, reactivation of the varicella-zoster virus can cause chickenpox in people who haven't previously had chickenpox or been vaccinated for it. (harbouchanews.com)
- A unique type of labyrinthitis may be caused by reactivation of the varicella-zoster virus, called Ramsay Hunt syndrome , or herpes zoster oticus. (ent-hns.net)
Chickenpox4
- Chickenpox Chickenpox is a highly contagious viral infection with the varicella-zoster virus that causes a characteristic itchy rash, consisting of small, raised, blistered, or crusted spots. (msdmanuals.com)
- Early in your childhood, you may have chickenpox because of the varicella-zoster virus . (allthingsmedicine.com)
- Also, one can have a vaccination for chickenpox to prevent the varicella-zoster virus reactivation. (allthingsmedicine.com)
- The varicella-zoster virus lies dormant in your nerve cells after you have chickenpox. (shinglestalk.com)
Infection11
- Herpes zoster oticus (HZ oticus), also known as Ramsay Hunt syndrome, is a viral infection of the inner, middle, and external ear caused by spread of the varicella-zoster virus to the facial nerves. (medscape.com)
- An additional complication of herpes zoster viral infection is postherpetic neuralgia . (medscape.com)
- 10%) give a history of previous herpes zoster viral infection. (medscape.com)
- Herpes zoster oticus is a herpes zoster virus infection of the clusters of nerve cells (ganglia) that control the nerves responsible for hearing and balance (8th cranial nerve) and for facial movement (7th cranial nerve). (msdmanuals.com)
- Herpes zoster oticus (HZ oticus) is a viral infection of the inner, middle, and external ear. (medscape.com)
- Intraoral herpes zoster closely resembles recurrent HHV-1 infection, but the lesions generally follow a dermatome and stop sharply at the midline, as shown here. (medscape.com)
- The cause of this disorder is reactivation of a latent varicella-zoster virus infection occurring years after the primary infection. (medscape.com)
- Ramsay Hunt Syndrome is a phrase used in neurology for few different disease conditions, but on majority occasions it means facial nerve (7th cranial nerve) paralysis due to infection with a herpes family virus called as Varicella Zoster. (your-neurologist.com)
- The acute infection with Varicella Zoster virus causes the familiar chicken pox. (your-neurologist.com)
- It occurs as a result of reactivation oflatent varicella zoster virus infection in the geniculate ganglion of the facial nerve. (org.pk)
- In addition to herpes zoster infection in any region of the head or neck, the development of facial paralysis is the second most common cause of the disease and the addition of the 8th nerve involvement symptoms is the third form of the disease. (org.pk)
Symptoms5
- Doctors may give people medications to relieve their symptoms and treat herpes zoster oticus. (msdmanuals.com)
- [ 2 ] Auditory and vestibular symptoms develop in approximately 25% of patients with herpes oticus, in addition to the facial paralysis and vesicular rash that characterize the disease. (medscape.com)
- The incidence of cochlear symptoms in herpes zoster oticus was not related to the severity of facial paralysis. (jebmh.com)
- Herpes zoster oticus show varying signs and symptoms of damage among individuals. (allthingsmedicine.com)
- Patients with herpes zoster forms a major fraction of patients in dermatology department, since this condition can cause various complications, such as ocular complications, Ramsay Hunt syndrome, and postherpetic neuralgia during or after treatment, and also indicate prodro- mal symptoms of malignant tumors and immunodeficiency 4 . (e-jmi.org)
Viral1
- A unique form of viral labyrinthitis is herpes zoster oticus, or Ramsay-Hunt syndrome. (medscape.com)
Occurs1
- In general, facial herpes zoster occurs when the virus is transported through the cranial nerves. (e-jmi.org)
Acute1
- There is no direct evidence that aciclovir has a beneficial effect on outcomes in Ramsay Hunt syndrome (herpes zoster oticus, HZO, associated with acute facial paralysis), although the use of aciclovir in patients with HZ in other parts of the body suggest that they have a role also in HZO. (unboundmedicine.com)
Rash3
- The strict definition of the Ramsay Hunt syndrome is peripheral facial nerve palsy accompanied by an erythematous vesicular rash on the ear (zoster oticus) or in the mouth. (juniperpublishers.com)
- If rash is seen then the fluid inside the rash can be sent to lab for microscopic examination to demonstrate characteristic herpes inclusion bodies. (your-neurologist.com)
- Herpes zoster oticus (Ramsay Hunt Syndrome) is characterized by facial nerve paralysis, ear pain and auricular skin rash. (org.pk)
Known as herpes1
- Ramsay Hunt syndrome , also known as herpes zoster oticus, is a rare yet severe neurological disorder characterized by facial paralysis. (kidy-kidy.com)
Vestibular2
- Herpes zoster oticus involving vestibular nerve without facial nerve involvement is uncommon and is only reported in a limited body of literature. (ejao.org)
- Evidence of inflammation within the auditory and vestibular nerves and the labyrinth has been noted on post-mortem examinations of patients with herpes zoster oticus [ 6 ]. (juniperpublishers.com)
Latent3
- Herpes zoster (HZ) oticus is caused by the reactivation of latent varicella-zoster virus (VZV) that has remained dormant within sensory ganglia (commonly the geniculate ganglion) of the facial nerve. (medscape.com)
- Pain in nerves, frequently involving facial SKIN , resulting from the activation the latent varicella-zoster virus ( HERPESVIRUS 3, HUMAN ). (nih.gov)
- Herpes zoster is caused by the reactivation of varicella-zoster virus (VZV), which belongs to the human herpesvirus family and remains latent in the sensory nerve ganglia after an earlier attack of varicella. (e-jmi.org)
Postherpetic3
- We assessed the proportion of ocular complications, Ramsay-Hunt syndrome, and postherpetic neuralgia in patients with facial herpes zoster according to several clinical factors including age, sex, dermatomes, underlying disease, and time to initiate antiviral treatment. (e-jmi.org)
- These results demonstrated that facial herpes zoster was associated with ocular complications, Ramsay-Hunt syndrome, and postherpetic neuralgia. (e-jmi.org)
- Herein, we present a retrospective study on clinical presentations and risk factors for ocular complications, Ramsay Hunt syndrome, and postherpetic neuralgia comprising 676 patients hospitalized for facial herpes zoster in the department of dermatology at the Dongguk University Gyeongju Hospital during a 10-year period. (e-jmi.org)
Pathogenesis4
- ABSTRACT: OBJECTIVES: Pure tone audiometry and caloric test in patients with herpes zoster oticus were performed to determine the biologic features of the varicella zoster virus (VZV) and the pathogenesis of vestibulocochlear nerve disease in herpes zoster oticus. (jebmh.com)
- Navneet G, Reeta G, Abhaya L, Kalu R, Aquib M D. The Pathogenesis of Vestibule Cochlear Nerve Disease in Herpes Zoster Oticus. (juniperpublishers.com)
- Audio logical assessment in patients with herpes zoster oticus were performed to determine the biologic features of the varicella zoster virus (VZV) and the pathogenesis of vestibule cochlear nerve disease in herpes zoster oticus. (juniperpublishers.com)
- The findings were compared with the clinical severity of facial paralysis and patient age to clarify the pathogenesis of vestibule cochlear nerve disease in herpes zoster oticus. (juniperpublishers.com)
Disorders2
- Available at: https://www.ninds.nih.gov/Disorders/All-Disorders/Herpes-Zoster-Oticus-Information-Page. (epnet.com)
- In patients with herpes zoster oticus, disorders of the eighth cranial nerve are frequently observed. (juniperpublishers.com)
Neurological2
- But these herpes group of viruses have a tendency to live in a dormant manner in the body for long time and when an opportunity strikes like for example when the body immunity weakens due to any reason they get reactivated and cause some neurological disease. (your-neurologist.com)
- Since facial herpes zoster may leave neurological sequelae, rapid diagnosis and prompt initiation of treatment are most important 13 . (e-jmi.org)
Cranial polyneuropathy3
- Herpes Zoster Oticus with Cranial Polyneuropathy without Involvement of Facial Nerve. (ejao.org)
- Differentiation of Ramsay Hunt syndrome or herpes zoster oticus associated with cranial polyneuropathy from other diseases is essential for accurate diagnosis and proper management. (ejao.org)
- Ramsay Hunt syndrome is a cranial polyneuropathy caused by reactivation of varicella zoster viruses. (org.pk)
Trigeminal Nerve1
- Reactivation of herpes zoster involving the trigeminal nerve may mimic odontogenic pain during the prodromal stage of the disease. (drjack.world)
Auricular1
- The most notable type of Ramsay Hunt syndrome is Ramsay Hunt syndrome type 2 (sometimes referred to as herpes zoster oticus or auricular herpes zoster). (allaboutvision.com)
Https1
- Available at: https://www.dynamed.com/condition/herpes-zoster. (epnet.com)
Ramsay Hunt13
- Also, in patients with Ramsay Hunt syndrome, vesicles may appear before, during, or after facial palsy (zoster sine herpete). (medscape.com)
- In the head and neck region, the most common presentation of herpes zoster is Ramsay Hunt syndrome which involves the neurons in the geniculate ganglion of the cranial nerve (CN) VII. (ejao.org)
- However, Ramsay Hunt syndrome has a known cause (varicella-zoster virus), whereas Bell's palsy does not have an exact origin. (allaboutvision.com)
- The varicella-zoster virus causes Ramsay Hunt syndrome. (allaboutvision.com)
- Cellulitis , Perichondritis , Herpes zoster oticus ( Ramsay Hunt syndrome ) [16] . (wikidoc.org)
- Ramsay Hunt syndrome also termed Hunt's Syndrome and herpes zoster oticus. (healthhyme.com)
- We also refer to Ramsay Hunt syndrome as Facial nerve palsy or herpes zoster oticus. (allthingsmedicine.com)
- Antiviral therapy for Ramsay Hunt syndrome (herpes zoster oticus with facial palsy) in adults. (unboundmedicine.com)
- Ramsay Hunt syndrome related with the facial nerve is also called as herpes zoster oticus. (your-neurologist.com)
- Complications of herpes zoster oticus and Ramsay Hunt syndrome might include hearing loss and facial weakness. (shinglestalk.com)
- Herpes zoster, cranial nerve, ramsay hunt. (org.pk)
- Herpes zoster oticus (HZO) or Ramsay Hunt Syndrome (RHS) is one of the rare causes of facial paralysis and accounts for 3% to 12% of patients with facial paralysis. (org.pk)
- 1-4 Ramsay Hunt syndrome, which has a prevalence of 1% in herpes zoster infections, was first described by Ramsay Hunt in 1907. (org.pk)
Encephalitis1
- Zoster encephalitis is also rare but is reported in otherwise healthy individuals. (medscape.com)
Recurrent1
- With a review of literature, we report a case of herpes zoster oticus with selective involvement of CN VIII, IX and X. Interestingly, the motor fibers of CN VII were spared while ipsilateral recurrent laryngeal nerve involvement was evident. (ejao.org)
Affects2
- Herpes zoster oticus is caused by herpetic viruses including varicella zoster and most commonly affects cranial nerves (CN) VII and VIII. (ejao.org)
- The condition develops when the varicella-zoster virus affects the facial nerve near the inner ear. (allaboutvision.com)
Facial nerves1
- Anytime in your life, the Varicella-Zoster virus reactivates in your facial nerves, which will lead to a condition of facial paralysis or facial palsy. (allthingsmedicine.com)
Intraoral1
- Intraoral zoster may be preceded by facial pain, toothache, or intraoral tingling. (drjack.world)
Vaccine1
- This risk of this problem may be lowered by getting the herpes zoster vaccine . (epnet.com)
Herpetic1
- Herpes zoster resulting from reactivation of preexisting Varicella zoster virus (VZV) is characterized by unilateral painful herpetic vesicular eruption along the dermatome. (ejao.org)
Vesicular eruption1
- HZ oticus manifests as severe otalgia and associated cutaneous vesicular eruption, usually of the external canal and pinna. (medscape.com)
Diagnosis2
- Doctors typically base the diagnosis of herpes zoster oticus on results of a physical examination. (msdmanuals.com)
- Laboratory testing of affected tissue or fluid from the vesicles is needed for a conclusive herpes zoster diagnosis. (drjack.world)
Disease1
- STUDY DESIGN: A retrospective chart review of 160 patients with herpes zoster oticus was designed in order to determine the classic characteristics of vestibulocochlear nerve disease associated with the syndrome. (jebmh.com)
Unilateral1
- For example, zoster duplex unilateralis refers to the involvement of two unilateral dermatomes. (medscape.com)
Antiviral1
- To decrease the risk of complications associated with facial herpes zoster, patients should receive appropriate antiviral therapy and interdepartmental consultations immediately. (e-jmi.org)
Clinical Features1
- This study aimed to investigate the clinical features and risk factors with facial herpes zoster. (e-jmi.org)
Dermatomes1
- Cases of zoster simultaneously occurring in seven noncontiguous dermatomes have been reported. (medscape.com)
Condition1
- The condition is also called Herpes Zoster Oticus. (webnewsobserver.com)
Presentation1
- We report a rare presentation of herpes zoster occurring over hard palate. (drjack.world)
Characteristic1
- You may see characteristic herpes vesicular eruptions in the outer ear. (your-neurologist.com)
Virus5
- RHS is caused by the varicella zoster virus (VZV). (epnet.com)
- It is caused by the varicella-zoster virus (VZV). (medlineplus.gov)
- Cases of actual monoplegia due to varicella-zoster virus (VZV) brachial plexus neuritis have been reported. (medscape.com)
- You can expect huskiness, dry and irritated eyes, and vertigo because of the damage caused by the varicella-zoster virus. (allthingsmedicine.com)
- Caused by Herpes zoster virus resulting in facial nerve damage. (allthingsmedicine.com)
Patients7
- The incidence rates of HZ oticus in males and females are equal, and incidence increases significantly in patients older than 60 years. (medscape.com)
- This is more common in immunosuppressed patients who use acyclovir (or other agents) as zoster prophylaxis. (medscape.com)
- also reported that the presence of VZV in the middle ear mucosa of patients with herpes zoster oticus suggested the arrival of VZV in the labyrinth through dehiscence of the facial nerve canal, creating a passage through the oval and/or round window [ 7 ]. (juniperpublishers.com)
- This study focused on the audio logical findings by pure tone audiometry and impedance audiometry of patients with herpes zoster oticus. (juniperpublishers.com)
- To establish a relationship between adverse affect of Herpes Zoster Octicus and Hearing system, we collected data of pure tone audiometry (PTA) and Impedance Audiometry of 60 patients with herpes zoster octicus, who had visited ASCOMS for a hearing check up over past 2 years. (juniperpublishers.com)
- We examined the medical records of 676 patients with facial herpes zoster during a 10-year-period from 2011 to 2020. (e-jmi.org)
- This study included patients who were hospitalized for facial herpes zoster at our department of dermatology. (e-jmi.org)