An abnormal extension of a gingival sulcus not accompanied by the apical migration of the epithelial attachment.
Surgical dressings, after periodontal surgery, applied to the necks of teeth and the adjacent tissue to cover and protect the surgical wound. (Boucher's Clinical Dental Terminology, 4th ed, p218)
Oral tissue surrounding and attached to TEETH.
Excessive growth of the gingiva either by an increase in the size of the constituent cells (GINGIVAL HYPERTROPHY) or by an increase in their number (GINGIVAL HYPERPLASIA). (From Jablonski's Dictionary of Dentistry, 1992, p574)
'Gingival diseases' is a general term for conditions affecting the soft tissues surrounding and supporting the teeth, primarily characterized by inflammation, bleeding, redness, or swelling, which can progress to periodontal disease if left untreated.
Non-inflammatory enlargement of the gingivae produced by factors other than local irritation. It is characteristically due to an increase in the number of cells. (From Jablonski's Dictionary of Dentistry, 1992, p400)
Generalized or localized diffuse fibrous overgrowth of the gingival tissue, usually transmitted as an autosomal dominant trait, but some cases are idiopathic and others produced by drugs. The enlarged gingiva is pink, firm, and has a leather-like consistency with a minutely pebbled surface and in severe cases the teeth are almost completely covered and the enlargement projects into the oral vestibule. (Dorland, 28th ed)
Inflammation of gum tissue (GINGIVA) without loss of connective tissue.
'Gingival neoplasms' are abnormal, uncontrolled growths of tissue originating from the gingiva, which can be benign (non-cancerous) or malignant (cancerous), often manifesting as swellings, ulcerations, or masses within the oral cavity.

Discrete proteolysis of focal contact and adherens junction components in Porphyromonas gingivalis-infected oral keratinocytes: a strategy for cell adhesion and migration disabling. (1/15)

Adhesive interactions of cells are critical to tissue integrity. We show that infection with Porphyromonas gingivalis, a major pathogen in the periodontal disease periodontitis, interferes with both cell-matrix and cell-cell adhesion in the oral keratinocyte cell line HOK-16. Thus, infected cells showed reduced adhesion to extracellular matrix, changes in morphology from spread to rounded, and impaired motility on purified matrices in Transwell migration assays and scratch assays. Western blot analysis of P. gingivalis-challenged HOK-16 cells revealed proteolysis of focal contact components (e.g., focal adhesion kinase), adherens junction proteins (e.g., catenins), and adhesion signaling molecules (e.g., the tyrosine kinase SRC). Proteolysis was selective, since important components of adherens junctions (E-cadherin) or signaling molecules (extracellular signal-regulated kinases ERK1/2) were not degraded. The virulence factors gingipains, cysteine proteinases expressed by P. gingivalis, are likely responsible for this proteolytic attack, since they directly digested specific proteins in pull-down experiments, and their proteolytic activity was blocked by the cysteine proteinase inhibitor N-alpha-p-tosyl-L-lysine chloromethyl ketone and also by a caspase inhibitor. Proteolysis was strain dependent, such that ATCC 33277 and 381 had high proteolytic potential, whereas W50 showed almost no proteolytic activity. These findings may help explain the formation of gingival pockets between cementum and periodontal epithelium, a hallmark of periodontitis. Furthermore, they illustrate a new pathogenetic paradigm of infection whereby bacteria may disrupt the integrity of epithelia.  (+info)

Evaluation of antibacterial activity of Mangifera indica on anaerobic dental microglora based on in vivo studies. (2/15)

The present study was carried out to compare the subgingival plaque microflora in mango leaf users. Fifty subjects of both sexes, 25 of them used tooth brush and 25 used mango leaf as their home care hygiene device were included in the study. The microbiological evaluation for specific bacterial counts of Actinobacillus actinomycetemcomitans, Prevotella intermedia, Porphyromonas gingivalis, Fusobacterium nucleatum and Peptostreptococcus micros were carried out for all subject. Specific microbial evaluation revealed significant decrease in the proportion of P. intermedia and P. gingivalis in mango leaf users compared to tooth brush users. It shows that mangiferin possesses antibacterial activity in vivo against specific periodontal pathogens such as P. intermedia and P. gingivalis. Use of mango leaf in conjunction with a tooth brush will be a good home care device for maintenance of oral hygiene.  (+info)

Superinfecting microorganisms in patients under treatment with cyclosporin-A and its correlation to gingival overgrowth. (3/15)

The aim of this study was to identify the presence of superinfecting microorganisms (Gram-negative enteric rods and Candida sp.) in heart transplant patients and correlate this with gingival overgrowth. Thirty patients (10 females, 20 males--mean age 45 years) were examined. All were under cyclosporin-A (CsA) therapy. Patients who had taken any antibiotics 3 months prior the study or had been submitted to periodontal therapy were not enrolled. Patients were required to have at least 6 teeth. The plaque index (PI), gingival index (GI), pocket depth (PD) and clinical attachment level (CAL) were recorded. Microbiological samples were taken from sulcus/pocket (s/p) and from stimulated saliva (ss) and submitted to analysis. Patients were divided into two groups: the ones with gingival overgrowth (GO) and those without gingival overgrowth (WGO). After statistical analysis (chi-square test, Student's t-test, Fisher test, p < or = 0.05), we concluded that there was no statistical difference between groups in the parameters of gender, CsA dosage, time since transplantation, PI, GI, PD and CAL. Gram-negative rods from either the sulcus/pocket or saliva samples were not found. Candida sp. was detected (s/p-30% and ss-30%). Stimulated saliva samples analysis determined that the presence of Candida sp. was associated with patients without gingival overgrowth.  (+info)

Evaluation of the marginal gingival health using laser Doppler flowmetry. (4/15)

The purposes of this study were to compare the gingival blood flow (GBF) in test sites (teeth retaining fixed partial dentures) and control sites (contralateral natural teeth) and investigate whether there is any relationship between clinical indices and GBF values. Twelve healthy subjects (6 females and 6 males) aged 20 to 54 years were enrolled this study. The GBF was measured from the middle point of the marginal gingiva in the test and control sites using laser Doppler flowmetry (LDF). Additionally, plaque index, gingival index and probing depth measurements were recorded. Statistically significant difference (p<0.05) was found between the test and control sites for marginal GBF. In contrast, no significant difference (p>0.05) was found between test and control sites with respect to the clinical indices, except for plaque index. The findings of this study suggest that there is a significant relation between resin-bonded fixed partial dentures with margins located subgingivally and marginal GBF. Clinical indices are helpful to collect information about the clinical health status of gingival tissues, but GBF is a good tool to measure gingival tissue blood flow and assess periodontal health. In conclusion, laser Doppler flowmetry can be used together with clinical indices to evaluate the marginal gingival health.  (+info)

Osteocalcin in serum, saliva and gingival crevicular fluid: their relation with periodontal treatment outcome in postmenopausal women. (5/15)

BACKGROUND: Osteocalcin levels have been postulated as a marker of inhibition of bone formation. The aim of the present study was to assess plasma, saliva and GCF levels of osteocalcin and correlate them with periodontal treatment outcome in postmenopausal women. METHODS: Thirty-nine postmenopausal women (57.8 -/+8.5 years old) were recruited for the study. Periodontal examination of all women was carried out and plaque, bleeding on probing, probing depth (PD), and clinical attachment level (CAL) were recorded. Serum, saliva and gingival crevicular fluid osteocalcin were measured. Then, periodontal treatment was carried out. Six months after the first appointment a second periodontal examination was carried out. RESULTS: Mean PD and mean CAL decreased significantly at second appointment in the group with serum osteocalcin concentration <10 ng/ml (15.8 -/+15.8% and 15.3 -/+ 21.2% respectively; p < 0.05). Mean PD decreased significantly at second appointment in the groups with saliva osteocalcin concentration < 3 ng/ml (17.1 -/+ 15.9%; p < 0.05) and 3-7 ng/ml (16.2 -/+18.1%; p < 0.05). CONCLUSIONS: Low serum osteocalcin concentration is associated to a significantly higher percentage of decrease in PD and CAL after periodontal treatment in postmenopausal women. Low saliva osteocalcin concentrations are significantly associated to a higher percentage of decrease in PD.  (+info)

Lactobacillus uli sp. nov. and Lactobacillus rimae sp. nov. from the human gingival crevice and emended descriptions of lactobacillus minutus and Streptococcus parvulus. (6/15)

Lactobacillus uli sp. nov. and Lactobacillus rimae sp. nov. are described. These organisms are short, gram-positive, strictly anaerobic, rod-shaped bacteria that have DNA G+C contents of 53 and 45 mol%, respectively, produce major amounts of lactic acid, and have been isolated from human gingival crevices and periodontal pockets. The major cellular fatty acid derivatives for both species are C18:1 cis-9 fatty acid methyl ester and C18:1 cis-9 dimethylacetyl. The type strain of L. uli is strain VPI D76D-27C (= ATCC 49627), and the type strain of L. rimae is strain D140H-11A (= ATCC 49626). Emended descriptions of Lactobacillus minutus (based on selected strains) and Streptococcus parvulus (based on many additional strains) also are given.  (+info)

Subgingival microbiota in squirrel monkeys with naturally occurring periodontal diseases. (7/15)

The squirrel monkey (Saimiri sciureus) has been proposed as an in vivo model for the study of subgingival colonization by suspected periodontopathogens, such as black-pigmented porphyromonads and prevotellas (BP/P). However, the indigenous microbiota of the squirrel monkey has not been well described. Therefore, in order to more fully characterize the oral microbiota of these animals, we studied two groups of squirrel monkeys from widely different sources. Group I consisted of 50 breeding colony monkeys ranging in age from 9 months to over 6 years which had been raised in captivity; group II consisted of 16 young sexually mature monkeys recently captured in the wild in Guyana. Group I animals in captivity had developed moderate to severe gingivitis, with a mean gingival index (GI) of 2.6; 52% of the sites bled, 26% had detectable calculus, and 83% had detectable BP/P. A group I subset (six animals), for which predominant cultivable microbiota was described, had a mean GI of 2.4. Colony morphology enumeration revealed that five of the six subset animals were detectably colonized with BP/P (range, 0 to 16.9%) and Actinobacillus actinomycetemcomitans (range, 0 to 3.9%); all subset animals were colonized with Fusobacterium species (range, 0.8 to 3.6%), Actinomyces species (range, 2.3 to 11%), and gram-positive cocci (range, 1.4 to 21.4%). Predominant cultivable microbiota results revealed the presence of many bacterial species commonly found in the human gingival sulcus. At baseline, group II animals were clinically healthy and had a mean GI of 1.4; 67% of the sites bled and 2.1% had calculus, and none of the animals had detectable BP/P. Neisseriae were very common in noninflamed sites. Subsequently, when inflamed sites were compared with noninflamed sites in group II animals after they had been maintained in captivity for 6 months, inflamed sites exhibited a more complex microbiota and increased proportions of gram-negative rods and asaccharolytic bacteria.  (+info)

Flies blown disease--oral myiasis. (8/15)

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A gingival pocket, also known as a sulcus, is a small space or groove between the gum tissue (gingiva) and the tooth. It's a normal anatomical structure found in healthy teeth and gums, and it measures about 1-3 millimeters in depth. The purpose of the gingival pocket is to allow for the movement of the gum tissue during functions such as eating, speaking, and swallowing.

However, when the gums become inflamed due to bacterial buildup (plaque) or other factors, the pocket can deepen, leading to the formation of a pathological gingival pocket. Pathological pockets are typically deeper than 3 millimeters and may indicate the presence of periodontal disease. These pockets can harbor harmful bacteria that can cause further damage to the gum tissue and bone supporting the tooth, potentially leading to tooth loss if left untreated.

Periodontal dressings are medicated, semi-rigid materials used in dentistry to protect and support traumatized or surgically treated periodontal tissues during the healing process. They act as a barrier, shielding the area from physical trauma, bacterial invasion, and food impaction, thereby promoting optimal healing. These dressings are typically applied after procedures such as deep scaling, root planing, or periodontal surgery.

Gingiva is the medical term for the soft tissue that surrounds the teeth and forms the margin of the dental groove, also known as the gum. It extends from the mucogingival junction to the base of the cervical third of the tooth root. The gingiva plays a crucial role in protecting and supporting the teeth and maintaining oral health by providing a barrier against microbial invasion and mechanical injury.

Gingival overgrowth, also known as gingival hyperplasia or hypertrophy, refers to an abnormal enlargement or growth of the gum tissue (gingiva) surrounding the teeth. This condition can be caused by various factors, including poor oral hygiene, certain medications (such as phenytoin, cyclosporine, and calcium channel blockers), genetic predisposition, and systemic conditions like vitamin C deficiency or leukemia.

Gingival overgrowth can lead to several complications, such as difficulty in maintaining oral hygiene, which may result in periodontal disease, tooth decay, bad breath, and potential loss of teeth. In some cases, the enlarged gum tissue may also cause discomfort or pain during speaking, chewing, or brushing. Treatment for gingival overgrowth typically involves improving oral hygiene, adjusting medications if possible, and undergoing surgical procedures to remove the excess gum tissue. Regular dental check-ups and cleanings are essential in managing and preventing this condition.

Gingival diseases are infections or inflammations that affect the gingiva, which is the part of the gum around the base of the teeth. These diseases can be caused by bacteria found in dental plaque and can lead to symptoms such as redness, swelling, bleeding, and receding gums. If left untreated, gingival diseases can progress to periodontal disease, a more serious condition that can result in tooth loss. Common types of gingival diseases include gingivitis and periodontitis.

Gingival hyperplasia is a condition characterized by an abnormal growth or enlargement of the gingiva (gum tissue). This condition can be caused by various factors, including bacterial infection, certain medications (such as phenytoin, cyclosporine, and nifedipine), systemic diseases (such as leukemia, vitamin C deficiency, and Crohn's disease), and genetic disorders.

The enlarged gum tissue can be uncomfortable, irritated, and prone to bleeding, especially during brushing or flossing. It may also make it difficult to maintain good oral hygiene, which can increase the risk of dental caries and periodontal disease. Treatment for gingival hyperplasia typically involves improving oral hygiene, controlling any underlying causes, and in some cases, surgical removal of the excess tissue.

Gingival fibromatosis is a benign (non-cancerous) condition characterized by the excessive growth of gum (gingival) tissue. The overgrowth can affect one or both the maxilla (upper jaw) and mandible (lower jaw) and can lead to various dental and oral health issues, such as difficulty in chewing, speaking, and maintaining proper oral hygiene.

The etiology of gingival fibromatosis can be divided into two categories: hereditary and acquired. Hereditary gingival fibromatosis is often associated with genetic mutations, while acquired gingival fibromatosis can result from factors like chronic inflammation due to poor oral hygiene, certain medications (such as phenytoin, cyclosporine, or nifedipine), and systemic conditions (like leukemia).

The management of gingival fibromatosis typically involves surgical removal of the excess tissue. However, recurrence is common due to the condition's tendency for regrowth. Regular follow-ups with a dental professional are essential to monitor any potential regrowth and maintain good oral hygiene.

Gingivitis is a mild form of gum disease (periodontal disease) that causes irritation, redness, swelling and bleeding of the gingiva, or gums. It's important to note that it is reversible with good oral hygiene and professional dental treatment. If left untreated, however, gingivitis can progress to a more severe form of gum disease known as periodontitis, which can result in tissue damage and eventual tooth loss.

Gingivitis is most commonly caused by the buildup of plaque, a sticky film of bacteria that constantly forms on our teeth. When not removed regularly through brushing and flossing, this plaque can harden into tartar, which is more difficult to remove and contributes to gum inflammation. Other factors like hormonal changes, poor nutrition, certain medications, smoking or a weakened immune system may also increase the risk of developing gingivitis.

Gingival neoplasms refer to abnormal growths or tumors that occur in the gingiva, which are the part of the gums that surround the teeth. These growths can be benign (non-cancerous) or malignant (cancerous). Benign neoplasms include conditions such as fibromas, papillomas, and hemangiomas, while malignant neoplasms are typically squamous cell carcinomas.

Gingival neoplasms can present with a variety of symptoms, including swelling, bleeding, pain, and loose teeth. They may also cause difficulty with chewing, speaking, or swallowing. The exact cause of these neoplasms is not always known, but risk factors include tobacco use, alcohol consumption, poor oral hygiene, and certain viral infections.

Diagnosis of gingival neoplasms typically involves a thorough clinical examination, including a dental exam and biopsy. Treatment options depend on the type and stage of the neoplasm, but may include surgery, radiation therapy, chemotherapy, or a combination of these approaches. Regular dental check-ups and good oral hygiene practices can help to detect gingival neoplasms at an early stage and improve treatment outcomes.

In a gingival pocket, no destruction of the connective tissue fibers (gingival fibers) or alveolar bone occurs. This early sign ... Gingival and periodontal pockets (also informally referred to as gum pockets) are dental terms indicating the presence of an ... the pocket is now lined by pocket epithelium (PE) instead of junctional epithelium (JE). To have a true periodontal pocket, a ... A periodontal pocket can become an infected space and may result in an abscess formation with a papule on the gingival surface ...
Gingival and periodontal pocketsGingival cyst of the adult • Gingival cyst of the newborn • Gingival enlargement • Gingival ... Free gingival margin • Frenulum linguae • Frey's syndrome • Fungiform papilla G. Walter Dittmar • Gardner's syndrome • Gargling ... fibers • Gingival sulcus • Gingivectomy • Gingivitis • Giovanni Battista Orsenigo • Glandular odontogenic cyst • Glasgow Dental ...
The difference between a gingival crevice and periodontal pocket is that former is 3mm. Periodontal pockets usually accompany ... the number of pockets, and whether the pockets bleed when examined with a dental probe. Indeed, VSC may themselves have been ... Gingival crevices are the small grooves between teeth and gums, and they are present in health, although they may become ... Their gums are evident with large pockets, where pus accumulation occurs. This nidus of infection can be a potential source for ...
A healthy gingival pocket will contain bacteria and some calculus kept in check by the immune system. As the pocket deepens, ... A variant of the periodontal abscess is the gingival abscess, which is limited to the gingival margin, has a quicker onset, and ... is a collection of pus that forms in the gingival crevices, usually as a result of chronic periodontitis where the pockets are ... 25 Apical abscesses can spread to involve periodontal pockets around a tooth, and periodontal pockets cause eventual pulp ...
It begins with the formation of plaque, specifically subgingival plaque within the gingival sulcus or periodontal pocket. This ... The probe is inserted into the gingival sulcus to measure its depth. Normal sulcus depth in the dog is < 3 mm and < 1 mm in ... They appear as an overgrowth of gingival or pulpal tissue. The lesions erode the dentin within a single tooth (or several ... Tromp, J. a. H.; Jansen, J.; Pilot, T. (1986). "Gingival health and frequency of tooth brushing in the beagle dog model". ...
In addition, mouthwash solution can be injected into periodontal pockets using the sub-gingival nozzle, used as a nozzle. After ... In the case of hard-to-reach areas such as when using braces, in gum pockets, the angle of the nozzle can be changed. Dentists ... On the other hand, there is a risk that food debris will get into the tooth pockets due to improper use ("flushing the tooth ... Regular use of an oral irrigator is believed to improve gingival health. The devices may also provide easier cleaning for ...
Another potential cause occurs when a periodontal pocket is scaled incompletely. Following this procedure, the gingival cuff ... However, if the opening of a periodontal pocket becomes obstructed, as may occur if the pocket has become very deep (e.g. with ... A gingival retraction cord which is accidentally left in situ is an occasional cause of a periodontal abscess. Penetrating ... response occurs when bacteria invade and multiply within the soft tissue of the gingival crevice/periodontal pocket. A pus- ...
It is found in the mouth inside the gingival pocket biofilm near the base of the teeth, and in periodontal pockets. Entamoeba ... "Use of PCR to detect Entamoeba gingivalis in diseased gingival pockets and demonstrate its absence in healthy gingival sites". ... 2014). "Detection of the amoeba Entamoeba gingivalis in periodontal pockets". Parasite. 21: 30. doi:10.1051/parasite/2014029. ...
A periodontal pocket is a dental term indicating the presence of an abnormally deepened gingival sulcus. Joplin RE, Davis SM ( ... 3) The probe should be run around the gingival pockets and the highest score derived in each sextant derived should be recorded ... One effect of proteolysis is that the pH of the gingival pocket with periodontal disease will increase and becomes slightly ... The gingival sulcus is an area of potential space between a tooth and the surrounding gingival tissue and is lined by sulcular ...
... it can result in gingival inflammation with pocket formation, gingival recession and loss of alveolar bone crest height. In ... Options available are gingival retraction cord, Magic Foam cord, and ExpaSyl. Another method to expose the margins of a ... Occluso-gingival length or height of the crown preparation affects both resistance and retention. Generally, the taller the ... This is crucial as remnants of temporary cement left on the tooth surface can compromise gingival health and interfere with ...
Clinical signs of periodontal pockets include bluish-red, thickened gingiva, gingival bleeding, localized pain and in some ... Healing of periodontal pockets are shown by a reduction in pocket depth. Although pocket depths can be reduced by decreasing ... A suprabony pocket occurs when there is horizontal bone loss, the bottom of the pocket is coronal to the alveolar bone. An ... Periodontal pockets may occur from either coronal swelling or apical migration. Pockets that occur due to coronal swelling with ...
Similar across the world, dental care for teeth with caries, calculus or gingival and periodontal pockets were also of concern ... own pockets. Although cases of malnutrition has decreased overall, it still remains a concern, particularly due to health ... of its citizens have to pay out-of-pocket payments. There is also an inadequate ratio of doctors to patients. There are 8 ...
Pocket depths greater than 3 mm can also be a sign of gingival hyperplasia. The periodontal probe can also be used to measure ... The first marking visible above the pocket indicates the measurement of the pocket depth. It has been found that the average, ... The tip of the instrument is placed with light pressure of 10-20 grams into the gingival sulcus, which is an area of potential ... Its main function is to evaluate the depth of the pockets surrounding a tooth in order to determine the periodontium's overall ...
... reduced pocket depth, less increase in gingival recession and more gain in hard tissue probing at re-entry surgery. However ... Pockets greater than 3mm in depth are considered to be unhealthy. True pocket formation of 4 mm or more are specifically ... and gingival recession is the predominant lesion before 40 years, while periodontal pocketing is the principal mode of ... This may also be caused by heavy-handed brushing or with a stiff tooth brush.) Deep pockets between the teeth and the gums ( ...
The probe is "walked around" measuring the depth of the gingival crevices/periodontal pockets (the gap between the tooth and ...
A Cochrane review found that GTR had a greater effect on probing measures (including improved attachment gain, reduced pocket ... are dental surgical procedures that use barrier membranes to direct the growth of new bone and gingival tissue at sites with ... E.g. Emdogain which has been shown to significantly improve probing attachment levels (1.1mm) and periodontal pocket depth ... as an alternative to resective surgical procedures to reduce pocket depths. A barrier membrane is utilized in the GBR technique ...
Toothbrushing can only clean to a depth of about 1.5 mm inside the gingival pockets, but a sustained regime of plaque removal ... Cobb CM, Rodgers RL, Killoy WJ (March 1988). "Ultrastructural examination of human periodontal pockets following the use of an ... Strydonck DA, Slot DE, Velden U, Weijden F. "Effect of a chlorhexidine mouthrinse on plaque, gingival inflammation and staining ... "Essential oils compared to chlorhexidine with respect to plaque and parameters of gingival inflammation: a systematic review". ...
... of the teeth in the presence or absence of secondary epithelial proliferation and pocket formation or secondary gingival ... almost all cases did exhibit varying degrees of gingival inflammation. Periodontal disease in children. In Goldman HM, Cohen DW ...
... pyorrheal pockets, and the gingival margin around the gums. T. tenax trophozoites multiply by longitudinal binary fission. ... Tartar between the teeth and the gingival margin of the gums are the primary areas of the mouth that may also potentially ... Biofilm harvested from infested areas of the periodontal pockets can be mounted onto a slide; T. tenax, if present, will be ... Microscopic examination of tonsillar crypts and pyorrheal pockets of patients with T. tenax infections often yields the typical ...
... and in gingival crevices or periodontal pocket (below the gumline). The release of waste products from the bacteria living in ... Hereditary gingival fibromatosis is the main example of a genetic disease causing gingival lesions. There is fibrous ... Specifically, hereditary gingival fibromatosis is known to cause non-plaque-induced gingival lesions. However, sometimes, there ... Periodontal pocket formation Gingival ulceration and suppuration Destruction of the alveolar bone and periodontal ligament ...
Immediately apical to the base of the pocket, and coronal to the most coronal of the gingival fibers is the junctional ... Most importantly is the formation of pocket epithelium within the periodontal pocket, which is a histopathological ... which lines the gingival sulcus from the base to the free gingival margin, where it interfaces with the epithelium of the oral ... the probing depth of the gingival sulcus may be considerably different from the true histological gingival sulcus depth. The ...
Periodontal abscess: begins in a periodontal pocket (see: periodontal abscess) Gingival abscess: involving only the gum tissue ... If the tooth has pre-existing periodontal disease, with pockets and loss of alveolar bone height, it is more likely to be a ... Similarly, in a periodontal abscess pus most likely discharges via the periodontal pocket, whereas a periapical abscess ... if it is closer to the gingival margin, it is more likely to be a periodontal abscess. ...
Gingival curettage is a surgical procedure designed to remove the soft tissue lining of the periodontal pocket with a curet, ... by the removal of pocket lining and junctional epithelium. Since there is no evidence that gingival curettage has any ... leaving only a gingival connective tissue lining. ... Gingival curettage, as originally conceived, was designed to promote new ... This indicates that the dental community as a whole regards gingival curettage as a procedure with no clinical value. Curettage ...
... pocket reduction surgery is a periodontal surgery performed in order to reduce the probeable depth of the gingival sulcus ( ... Reducing the depths of the periodontal pockets eliminates an environment that is hospitable for the more virulent periodontal ... American Academy of Periodontology Pocket Depth Reduction (Periodontology). ... known as a periodontal pocket in disease) to allow for less plaque accumulation and greater access for hygiene. ...
The gingival pocket between the tooth and the gingival should be no deeper than 1-3mm to be considered healthy. There is also ... 81 Periodontal health and gingival health Clinical gingival health on an intact periodontium Clinical gingival health on a ... Leukocytes and neutrophils are the main cells that phagocytose bacteria found in the gingival crevice or pocket. They migrate ... The gingival sulcus is lined by a non-keratinised layer called the oral sulcular epithelium; it begins at the gingival margin ...
... or undergo some form of gingival surgery to access the depths of the pockets and perhaps even change the pocket depths so they ... the person is said to have a gingival pocket if no migration of the epithelial attachment has occurred or a periodontal pocket ... a 2-mm pocket or a 6-mm pocket. However, pockets are generally accepted as self-cleansable (at home, by the person, with a ... gingival disease and conditions Periodontal health and gingival health Clinical gingival health on an intact periodontium ...
... to create physiological gingival contours with the sole purpose of recontouring the gingiva in the absence of the pockets. ... Gingival and periodontal disease often produces deformities in the gingiva that are conducive to the accumulation of plaque and ... This is a procedure performed to eliminate periodontal pockets along with the reshaping as part of the technique. This ... The technique resembles that of the festooning of an artificial denture, which consists of tapering the gingival margin, ...
... a pocket formation or displacement of the marginal gingivae away from the tooth by mechanical, chemical, or surgical means. It ... Gingival retraction or gingival recession is when there is lateral movement of the gingival margin away from the tooth surface ... A retraction cord, although more damaging to the gingival tissues, has proven to displace gingival tissues more effectively and ... "Gingival retraction". Mondofacto medical dictionary. 5 March 2000. American Dental Association. (2007). "Gingival Recession: ...
... gingival recession MeSH C07.465.714.258.480 - gingivitis MeSH C07.465.714.258.480.360 - gingival pocket MeSH C07.465.714.258. ... gingival diseases MeSH C07.465.714.258.250 - gingival hemorrhage MeSH C07.465.714.258.409 - gingival neoplasms MeSH C07.465. ... gingival MeSH C07.465.714.258.428.250 - gingival hyperplasia MeSH C07.465.714.258.428.260 - gingival hypertrophy MeSH C07.465. ... periodontal pocket MeSH C07.465.714.533.800 - periodontitis, juvenile MeSH C07.465.714.804 - tooth loss MeSH C07.465.714.836 - ...
It relates to lesions limited to gingival tissue. Clinical evidence and research shows that periodontal pockets contain large ... The main site for T. denticola habitation in the oral cavity is the gingival crevice. These spirochetes attach to proteins ( ... rev., and identification of new spirochete isolates from periodontal pockets". International Journal of Systematic Bacteriology ... including fibronectin and collagen) of local gingival fibroblasts, binding to their plasma membrane. A 53-kDa surface protein ...

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