No data available that match "Fetal Diseases"

*  Fetal origin of childhood disease: Intrauterine growth restriction in term infants and risk for hypertension at 6 years of age ...

... Shankaran, S., Das, A., Bauer, C. R., Bada, H., Lester, B., Wright, L., ... Poole, W. (2006). Fetal origin of childhood disease ... which may be a marker for adult cardiovascular disease. ,br,,br,,br, ...

*  Fetal Umbilical Cord Blood (UCB) Transplant for Lysosomal Storage Diseases - Full Text View -

Fetal Umbilical Cord Blood (UCB) Transplant for Lysosomal Storage Diseases (IUHST-001). This study has been withdrawn prior to ... Lysosomal Storage Diseases. Inborn Errors of Metabolism. Krabbe. Infantile metachromatic leukodystrophy disease. MLD. Tay Sachs ... The fetal weight will be calculated from formula: Y (kg) = (2.9026 x 10-1) (X 2.6528). The estimated fetal weight at that ... Treatment of Early Infantile-Onset Lysosomal Storage Diseases With Fetal Umbilical Cord Blood (UCB) Transplantation. ...

*  Thyroid Foundation of Canada

Fetal Thryoid Disease. Antithyroid medications, nonradioactive iodine and, very rarely, maternal thyroid antibodies can all ... Health Guides on Thyroid Disease Thyroid Disease: Know the Facts Thyroid Disease... Overview of thyroid function To Confirm the ... Since the mother is well, fetal thyrotoxicosis may not be suspected. Clues to the presence of fetal hyperthyroidism are fetal ... Thyroid Disease, Pregnancy & Fertility Thyroid Disease in Childhood Surgical Treatment of Thyroid Disease Thyroid Cancer Other ...

*  Cystic Lymphangioma disease: Malacards - Research Articles, Drugs, Genes, Clinical Trials

Global: Rare diseases Fetal diseases Cancer diseases Anatomical: Cardiovascular diseases Immune diseases See all MalaCards ... The MalaCards human disease database index: 1-9 A B C D E F G H I J K L M N O P Q R S T U V W X Y Z ... NIH Rare Diseases : 50 a cystic hygroma is a fluid-filled sac that results from a blockage in the lymphatic system. it is most ... Diseases related to Cystic Lymphangioma via text searches within MalaCards or GeneCards Suite gene sharing:. (show top 50) ( ...

*  ஆயுர்வேத மருத்துவம்: November 2010

Fetal diseases 22. Death Treatment of such evils: Diseases caused by the intake of viruddhahara can be cured by emesis, ... One who regularly takes butter milk never suffers from any disease and the diseases subdued by the use of butter milk, will ... 9. Plate made of palasa leaves: destroys gulma, vata diseases, colic, diseases due to aggravation, of kapha and pitta. ... skin diseases and poisoning, vertigo, dyspnoea, heart diseases. ... Kilasa type of skin disease 15. Obstinate skin diseases ...

*  Rapid Prototyping | Circulation

Use of casts in the necropsy diagnosis of fetal congenital heart disease. Br Heart J. 1992; 68: 481-484. ... A New Tool in Understanding and Treating Structural Heart Disease. Michael S. Kim, Adam R. Hansgen, Onno Wink, Robert A. Quaife ... First, congenital heart disease in children and adults has increased the appreciation of intracardiac shunt lesions and other ... Our mission is to build healthier lives, free of cardiovascular diseases and stroke. That single purpose drives all we do. The ...

*  Maternal obesity programs offspring nonalcoholic fatty liver disease by innate immune dysfunction in mice - Mouralidarane -...

The fetal and infant origins of disease. Eur J Clin Invest 1995; 25: 457-463.. Direct Link: ... Prevalence of nonalcoholic fatty liver disease and its association with cardiovascular disease among type 2 diabetic patients. ... Roles of liver innate immune cells in nonalcoholic fatty liver disease. World J Gastroenterol 2010; 16: 4652-4660.. *CrossRef, ... Nonalcoholic fatty liver disease. N Engl J Med 2002; 346: 1221-1231.. *CrossRef, ...

*  Saúde Pública - No cry at birth: global estimates of intrapartum stillbirths and intrapartum-related neonatal deaths No cry at...

Archives of Diseases in Childhood Fetal Neonatal Edition 1996;74:F56-9. ... Archives of Diseases in Childhood. Fetal Neonatal Edition 1996;74:F56-59. ... Classifying perinatal death: fetal and neonatal factors. British Journal of Obstetrics and Gynaecology 1986;93:1213-23. ... Epidemiology of fetal death in Latin America. Acta Obstetricia et Gynecologica Scandinavica 2000;79:371-8. ...

*  The long-term safety and efficacy of bilateral transplantation of human fetal striatal tissue in patients with mild to moderate...

Effect of fetal neural transplants in patients with Huntington's disease 6 years after surgery: a long-term follow-up study. ... Bilateral human fetal striatal transplantation in Huntington's disease. Neurology 2002;58:687-95. ... A patient with Huntington's disease and long-surviving fetal neural transplants that developed mass lesions. Acta Neuropathol ... Positron emission tomography after fetal transplantation in Huntington's disease. Ann Neurol 2005;58:331-7. ...

*  Epidemiology, management and outcome of ultrashort bowel syndrome in infancy | ADC Fetal & Neonatal Edition

Archives of Disease in Childhood - Fetal and Neonatal Edition 2017; 102 F469-F469 Published Online First: 21 Oct 2017. doi: ... Even in this small subgroup there is a spectrum of disease, at the worst end of the spectrum is the patient who has lost all ... Intestinal failure-associated liver disease in hospitalised children. Arch Dis Child 2012;97:211-4.doi:10.1136/archdischild- ... In the first year of life it is more likely related to underlying disease; this becomes very important with increasing number ...

*  Wiley Online Library: Search Results Page

Fetal nigral grafts survive and mediate clinical benefit in a patient with Parkinson's disease. Movement Disorders. Volume 13, ... Transplantation of fetal dopamine neurons in Parkinson's disease: One-year clinical and neurophysiological observations in two ... Functional fetal nigral grafts in a patient with Parkinson's disease: Chemoanatomic, ultrastructural, and metabolic studies. ... A double-blind controlled trial of bilateral fetal nigral transplantation in Parkinson's disease. Annals of Neurology. Volume ...

*  Indices of renal tubular function in perinatal asphyxia

Archives of Disease in Childhood -- Fetal & Neonatal Edition;Jul1997, Vol. 77 Issue 1, pF57 ... Fetal asphyctic preconditioning has been shown to modulate the cerebral inflammatory cytokine response, hereby protecting the ... Tubular injury in glomerular disease. Meyer, Timothy W. // Kidney International;Feb2003, Vol. 63 Issue 2, p774 Discusses the ... Cerebellar Cytokine Expression in a Rat Model for Fetal Asphyctic Preconditioning and Perinatal Asphyxia. Vlassaks, Evi; Brudek ...

*  Vaccinate or not vaccinate? - Page 2 - Homeschool World Forum

Disease Vaccine Name Manufacturer Cell line (Fetal) Polio Poliovax Aventis-Pasteur MRC-5 Measles, Mumps, Rubella Mumps-Rubella ... So, if the diseases were all declining, why do you think the gov't started requiring them? Hmmm....sounds to me like (and this ... Many diseases that were previously almost eradicated in the US are making a come back because of those no longer getting ... The diseases had been in decline long before the "required" vaccinations by our gov't. It's your choice to vaccinate your ...

*  Similar - Books on Google Play

It investigates the newly apparent fetal origins of allergic disease-that a mother's inflammatory response imprints on her ... In this compendium of the curious and fascinating organisms that cause disease, including Legionnaire's disease, mumps, CJD, ... In this compendium of the curious and fascinating organisms that cause disease, including Legionnaire's disease, mumps, CJD, ... An Epidemic of Absence: A New Way of Understanding Allergies and Autoimmune Diseases Moises Velasquez-Manoff September 4, 2012 ...

*  IL18 monoclonal antibody, clone SB116c - (MAB8223) - Products - Abnova

Fetal Diseases. *Fetal Membranes, Premature Rupture. *Gastrointestinal Neoplasms. *Genetic Predisposition to Disease ...

*  HSPA1A monoclonal antibody, clone 2A4 (ATTO 680) - (MAB11753) - Products - Abnova

Fetal Diseases. *Genetic Predisposition to Disease. *Glaucoma. *Glaucoma, Open-Angle. *Hearing. *Hearing Loss, Noise-Induced ...

*  VEGFA polyclonal antibody - (PAB18913) - Products - Abnova

Eye Diseases. *Familial Mediterranean Fever. *Familial Mediterranean fever. *Femur Head Necrosis. *Fetal Diseases ... Western blot analysis of human fetal skeletal muscle lysate with VEGFA polyclonal antibody (Cat # PAB18913) at 1 : 500 dilution ...

*  NFKBIB monoclonal antibody (M03), clone 3E11 - (H00004793-M03) - Products - Abnova

Fetal Diseases. *Genetic Predisposition to Disease. *Head and Neck Neoplasms. *HIV Infections ...

*  Alcohol intake and pregnancy

Centers for Disease Control and Prevention: Fetal alcohol spectrum disorders (FASDs) ... Fetal Alcohol Spectrum Disorders. Pediatrics 2015; 136:e1395.. *Carson G, Cox LV, Crane J, et al. Alcohol use and pregnancy ... Centers for Disease Control and Prevention (CDC). Alcohol use and binge drinking among women of childbearing age--United States ... The Centers for Disease Control and Prevention surveillance system reported that, within the month prior to being interviewed, ...

*  Most recent papers in the shared collection Lurie Cardiology Fellow Collection-Imaging | Read by QxMD

Diagnosis and treatment of fetal cardiac disease: a scientific statement from the American Heart Association. ... including the diagnosis and management of fetal cardiovascular disease. METHODS AND RESULTS: A writing group appointed by the ... ... magnetic-resonance-in-children-and-adults-with-congenital-heart-disease-scmr-expert-consensus-group-on-congenital-heart-disease ...

*  Similar - Books on Google Play

... with current and relevant information on everything from fetal origins of adult disease, to improving global maternal health, ... Master the ins and outs of a wide range of diseases, from common to rare, through detailed discussions of each disease s unique ... and surgery to help you effectively diagnose and treat fetal patients. Fetology considers the full implications of a fetal ... Drugs in Pregnancy and Lactation: A Reference Guide to Fetal and Neonatal Risk, Edition 10 Gerald G. Briggs June 10, 2014 2 ...

*  DMOZ - Health: Medicine: Medical Specialties: Perinatology: Research

Conducting studies into fetal and neonatal heart disease. Department of the Ud'A University of Chieti in Italy. ...

*  Johns Hopkins Medicine: Find a Doctor

Congenital Heart Disease, Congenital Heart Disease - Young Adult, Echocardiography, Fetal Cardiology, Heart Disease, Non- ... Expertise, Disease and Conditions: Cardiac Imaging, Cardiovascular Disease, Echocardiography, Ischemic Heart Disease, Non- ... Expertise, Disease and Conditions: Cardiomyopathy, Cardiovascular Disease in Women, Cardiovascular Diseases, Cardioversion, ... Expertise, Disease and Conditions: Adult Congenital Heart Disease, Cardiac Imaging, Cardiology, Cardiomyopathy, Cardiovascular ...

*  Light Exposure During Pregnancy Key to Normal Eye Development | UC San Francisco

Scientists say the unexpected finding offers a new basic understanding of fetal eye development and ocular diseases caused by ... is evidence supporting a new basic understanding of fetal eye development and potential eye diseases caused by the aberrant ... At UC San Francisco, we don't just treat diseases, we treat individuals. We put our patients' priorities at the center of our ... This has downstream effects on developing vasculature in the eye and is important because several major eye diseases are ...

*  Fetal Programming of Obesity - Full Text View -

Sexually transmitted diseases. *Medical complications during pregnancy. *Medications during pregnancy known to influence fetal ... Fetal Programming of Obesity. This study is ongoing, but not recruiting participants. ...

No data available that match "Fetal Diseases"

(1/1979) Increased phosphoglycerate kinase in the brains of patients with Down's syndrome but not with Alzheimer's disease.

Impaired glucose metabolism in Down's syndrome (DS) has been well-documented in vivo, although information on the underlying biochemical defect is limited and no biochemical studies on glucose handling enzymes have been carried out in the brain. Through gene hunting in fetal DS brain we found an overexpressed sequence homologous to the phosphoglycerate kinase (PGK) gene. This finding was studied further by investigating the activity levels of this key enzyme of carbohydrate metabolism in the brains of patients with DS. PGK activity was determined in five brain regions of nine patients with DS, nine patients with Alzheimer's disease and 14 controls. PGK activity was significantly elevated in the frontal, occipital and temporal lobe and in the cerebellum of patients with DS. PGK activity in corresponding brain regions of patients with Alzheimer's disease was comparable with controls. We conclude that our findings complement previously published data on impaired brain glucose metabolism in DS evaluated by positron emission tomography in clinical studies. Furthermore, we show that in DS, impaired glucose metabolism, represented by increased PGK activity, is a specific finding rather than a secondary phenomenon simply due to neurodegeneration or atrophy. These observations are also supported by data from subtractive hybridization, showing overexpressed PGK in DS brains at the transcriptional level early in life.  (+info)

(2/1979) Can transvaginal fetal biometry be considered a useful tool for early detection of skeletal dysplasias in high-risk patients?

OBJECTIVE: To evaluate the possibility of an early diagnosis of skeletal dysplasias in high-risk patients. METHODS: A total of 149 consecutive, uncomplicated singleton pregnancies at 9-13 weeks' amenorrhea, with certain menstrual history and regular cycles, were investigated with transvaginal ultrasound to establish the relationship between femur length and menstrual age, biparietal diameter and crown-rump length, using a polynomial regression model. A further eight patients with previous skeletal dysplasias in a total of 13 pregnancies were evaluated with serial examinations every 2 weeks from 10-11 weeks. RESULTS: A significant correlation between femur length and crown-rump length and biparietal diameter was found, whereas none was observed between femur length and menstrual age. Of the five cases with skeletal dysplasias, only two (one with recurrent osteogenesis imperfecta and one with recurrent achondrogenesis) were diagnosed in the first trimester. CONCLUSIONS: An early evaluation of fetal morphology in conjunction with the use of biometric charts of femur length against crown-rump length and femur length against biparietal diameter may be crucial for early diagnosis of severe skeletal dysplasias. By contrast, in less severe cases, biometric evaluation appears to be of no value for diagnosis.  (+info)

(3/1979) Comparison of prenatal ultrasound and postmortem findings in fetuses and infants with congenital heart defects.

OBJECTIVE: Detection of congenital heart defects by prenatal ultrasound examination has been one of the great challenges since the investigation for fetal anomalies became part of the routine fetal examination. This prospective study was designed to evaluate the concordance of prenatal ultrasound findings with autopsy examination in a population consisting of both referred women and non-selected pregnant women. DESIGN: Criteria for inclusion were an ultrasound examination at the National Center for Fetal Medicine and an autopsy performed during the years 1985-94. Results from the ultrasound and autopsy examinations were systematized into categories depending on the degree of concordance. RESULTS: Of 408 infants and fetuses with developmental anomalies, 106 (26%) had congenital heart defects. In 63 (59%) of these 106 cases, the heart defect was the principal reason for the termination of pregnancy or the cause of death. Excluding five cases with a secundum atrial septal defect, there was complete agreement between the ultrasound examination and the autopsy findings in 74 (73%) of 101 cases. In 18 cases, there were minor discrepancies between ultrasound and autopsy findings. The main diagnosis was thus correct in 92 cases (91%). From the first time period (1985-89) to the second (1990-94), the detection rate of all heart defects increased from 48% to 82%. CONCLUSION: This study confirms a good correlation between ultrasound and autopsy diagnoses in fetuses and infants with congenital heart defects. A significant improvement in the detection of heart defects occurred from the first time period to the second and was probably due to increased experience and technical advances.  (+info)

(4/1979) Diagnosis of twin reversed arterial perfusion sequence in the first trimester by transvaginal color Doppler ultrasound.

A case of twin reversed arterial perfusion (TRAP) sequence was diagnosed at 12 weeks' gestation using transvaginal color Doppler ultrasound, which demonstrated the presence of retrograde perfusion in the umbilical artery of the abnormal twin. Ultrasound imaging showed a monochorionic-diamniotic twin pregnancy with an inappropriately grown second twin, the morphological evaluation of which revealed an abnormal cephalic pole with acrania, diffuse subcutaneous edema and the presence of cardiac activity in an abnormal heart with a single chamber.  (+info)

(5/1979) Amylopectinosis in fetal and neonatal Quarter Horses.

Three Quarter Horses, a stillborn filly (horse No. 1), a female fetus aborted at approximately 6 months of gestation (horse No. 2), and a 1-month-old colt that had been weak at birth (horse No. 3), had myopathy characterized histologically by large spherical or ovoid inclusions in skeletal and cardiac myofibers. Smaller inclusions were also found in brain and spinal cord and in some cells of all other tissues examined. These inclusions were basophilic, red-purple after staining with periodic acid-Schiff (both before and after digestion with diastase), and moderately dark blue after staining with toluidine blue. The inclusions did not react when stained with Congo red. Staining with iodine ranged from pale blue to black. Their ultrastructural appearance varied from amorphous to somewhat filamentous. On the basis of staining characteristics and diastase resistance, we concluded that these inclusions contained amylopectin. A distinctly different kind of inclusion material was also present in skeletal muscle and tongue of horse Nos. 1 and 3. These inclusions were crystalline with a sharply defined ultrastructural periodicity. The crystals were eosinophilic and very dark blue when stained with toluidine blue but did not stain with iodine. Crystals sometimes occurred freely within the myofibers but more often were encased by deposits of amylopectin. This combination of histologic and ultrastructural features characterizes a previously unreported storage disease in fetal and neonatal Quarter Horses, with findings similar to those of glycogen storage disease type IV. We speculate that a severe inherited loss of glycogen brancher enzyme activity may be responsible for these findings. The relation of amylopectinosis to the death of the foals is unknown.  (+info)

(6/1979) Whence the arthrogrypotics?

During the course of a nation-wide survey of patients with bone and joint deformities, twenty-six individuals with arthrogryposis multiplex congenita, in the narrow and precise sense of the term, were investigated. No patient was more than twenty-four years of age. However, on a basis of the figures of population, it can be estimated that 21-0 +/- 6-5 older affected individuals should have been encountered. Furthermore, there was a relative excess of younger children. The series was reasonably unbiased, and as arthrogryposis is non-lethal the deficiency of affected adults is an anomalous finding. It is tentatively suggested that arthrogryposis might result from the intra-uterine influence of an unknown environmental agent which has been present in South Africa for only a limited period of time. Detection of this factor could be an important step in the prevention of the disease.  (+info)

(7/1979) Neurological morbidity after fetal supraventricular tachyarrhythmia.

BACKGROUND: Fetal tachyarrhythmia is a well-documented entity which, in the absence of pharmacological intervention, may lead to congestive heart failure, fetal hydrops and eventually fetal demise. The success rate of the implemented treatment is generally measured by survival and achievement of control of the arrhythmia. We report on the occurrence of associated cerebral damage in three patients with fetal tachycardia. METHODS: We describe three patients with a history of fetal supraventricular tachyarrhythmia who developed cerebral complications in utero. RESULTS: Two patients had cerebral hypoxic-ischemic lesions and one had hemorrhagic lesions present at birth. They had developed severe congestive heart failure and fetal hydrops secondary to fetal tachyarrhythmia, and there were no other obvious causes for the cerebral pathology. Two of these patients were referred to us antenatally. Therapy was instituted and resulted in control of the tachycardia and resolution of hydrops. The third patient was referred to our clinic shortly after birth because of severe circulatory problems secondary to fetal tachyarrhythmia. CONCLUSION: From these observations, we believe that a fetus with tachyarrhythmia and subsequent hydrops is at increased risk for the development of cerebral complications, due to the circulatory disturbances and sudden changes in heart rate which may lead to fluctuations in cerebral perfusion. This would imply that it is of the utmost importance to aim at immediate and complete control of the heart rate in the treatment of fetal tachyarrhythmia.  (+info)

(8/1979) Trisomy 10: first-trimester features on ultrasound, fetoscopy and postmortem of a case associated with increased nuchal translucency.

We report a case of the prenatal diagnosis of trisomy 10 in a fetus presenting with an increased nuchal translucency thickness (5 mm) on a routine first-trimester anomaly scan at 12 weeks' gestation. Multiple abnormalities were diagnosed by ultrasound and fetoscopy. Karyotyping on chorionic villus sampling led to the diagnosis of homogeneous trisomy 10 which was confirmed by in situ hybridization on fetal tissue samples. Postmortem examination confirmed major anatomical malformations, including facial cleft, arthrogryposis of the upper and lower limbs and bilateral diaphragmatic hernia, and also revealed hypoplastic lungs, right renal agenesis and a complex cardiac malformation. Trisomy 10 is an uncommon chromosomal abnormality that is likely to be associated with increased fetal nuchal translucency. This case also emphasizes the value of a detailed anomaly scan in high-risk patients in the first trimester of pregnancy.  (+info)


  • The disease is characterised pathologically by an early and progressive striatal neuronal cell loss and atrophy, which has provided the rationale for first clinical trials of neural repair using fetal striatal cell transplantation. (


  • Between 2000 and 2003, the 'NEST-UK' consortium carried out bilateral striatal transplants of human fetal striatal tissue in five HD patients. (


  • These diseases are known to cause severe and irreversible neurological disability in early infancy and which are lethal in childhood. (
  • The purpose of this study is to determine the safety of first trimester fetal stem cell therapy using unrelated donor partially HLA-matched stem and progenitor cells derived from human umbilical cord blood for the treatment of selected lysosomal storage diseases that are known to cause severe and irreversible neurological disability in early infancy and which are lethal in childhood. (


  • Conclusion In term infants, IUGR is linked to risk of hypertension in early childhood, which may be a marker for adult cardiovascular disease. (


  • The microbes in our bodies are part of our immune systems and protect us from disease. (


  • Both the gravida and her family benefit from factual, nonjudgmental information about the maternal and fetal risks of alcohol use and, if necessary, from counseling regarding strategies for cessation. (


  • Antibodies which damage the thyroid cells may result in lymphocytic thyroiditis (inflammation of the thyroid), also known as Hashimoto's disease. (

autosomal dominant

  • Huntington's disease (HD) is a fatal autosomal dominant neurodegenerative disease involving progressive motor, cognitive and behavioural decline, leading to death approximately 20 years after motor onset. (
  • Left ventricular noncompaction (LVNC) is a cardiomyopathy associated with sporadic or familial disease, the latter having an autosomal dominant mode of transmission. (

Liver Disease

  • Promoting adaptation is vital to improving long-term survival and can be achieved by optimising feeds, reducing intestinal failure liver disease and catheter-related bloodstream infections. (


  • Western blot analysis of human fetal skeletal muscle lysate with VEGFA polyclonal antibody (Cat # PAB18913) at 1 : 500 dilution. (


  • First, congenital heart disease in children and adults has increased the appreciation of intracardiac shunt lesions and other anatomy-based lesions. (
  • In the last several years, there has been a dramatic increase in the number of both approved and investigative catheter-based therapies for congenital as well as acquired structural heart disease that involve both modification of structures and implantation of novel devices. (
  • Cardiovascular magnetic resonance (CMR) has taken on an increasingly important role in the diagnostic evaluation and pre-procedural planning for patients with congenital heart disease. (
  • This article provides guidelines for the performance of CMR in children and adults with congenital heart disease. (


  • Cerebellar Cytokine Expression in a Rat Model for Fetal Asphyctic Preconditioning and Perinatal Asphyxia. (


  • Thyroid disease should be considered in patients undergoing investigation for menstrual problems or infertility. (
  • Specifically, the technology has been invaluable in medical disciplines in which disease management relies heavily on a thorough understanding of complex anatomic structures and has been shown to improve diagnostic accuracy, assist in preoperative planning, and enhance the teaching of patients, students, and physicians. (
  • No significant differences were found over time between the patients, grafted and non-grafted, on any subscore of the Unified Huntington's Disease Rating Scale, nor on the Mini Mental State Examination. (


  • Chickenpox is not what I'd call a dangerous disease unless you catch it when you're an older adult. (


  • This study concludes that fetal striatal allografting in HD is safe. (


  • Bacteria provide squid with invisibility cloaks, help beetles to bring down forests, and allow worms to cause diseases that afflict millions of people. (


  • Safety and feasibility of fetal intrap. (
  • It is designed to asses the safety and feasibility of administration of ALD-601 UCB cells to first trimester fetuses known to be affected with a lethal lysosomal storage disease. (
  • The first portion addresses preparation for the examination and safety issues, the second describes the primary techniques used in an examination, and the third provides disease-specific protocols. (


  • Thyroid disease is present in 2-5 percent of all women and 1-2 percent of women in the reproductive age group. (
  • Current 2-dimensional imaging techniques remain limited both in their ability to represent the complex 3-dimensional relationships present in structural heart disease and in their capacity to adequately facilitate often complex corrective procedures. (
  • Current 2-dimensional (2D) imaging techniques, however, fall short both in representing the complex 3D relationships present in structural heart disease and adequately facilitating often complex corrective procedures. (


  • Ebola, SARS, Hendra, AIDS, and countless other deadly viruses all have one thing in common: the bugs that transmit these diseases all originate in wild animals and pass to humans by a process called spillover. (


  • So, if the diseases were all declining, why do you think the gov't started requiring them? (