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  • carcinoma-in-s
  • The pattern of growth of TCCs can be papillary, sessile, or carcinoma-in-situ (CIS). (wikipedia.org)
  • Transitional cell papilloma, NOS M8120/2 Transitional cell carcinoma in situ Urothelial carcinoma in situ M8120/3 Transitional cell carcinoma, NOS Urothelial carcinoma, NOS Transitional carcinoma M8121/0 Schneiderian papilloma, NOS (C30.0, C31. (wikipedia.org)
  • hybridization
  • Diagnosis may include a fluorescence in situ hybridization (FISH) test, computed tomography urography (CTU), magnetic resonance urography (MRU), intravenous pyelography (IVP) x-ray, ureteroscopy, or biopsy. (wikipedia.org)
  • Myeloproliferative
  • Other genetic abnormalities in PDGFRB lead to various forms of potentially malignant bone marrow disorders: small deletions in and chromosome translocations causing fusions between PDGFRB and anyone of at least 30 genes can cause Myeloproliferative neoplasms that commonly involve eosinophilia, eosinophil-induced organ injury, and possible progression to aggressive leukemia (see blow). (wikipedia.org)
  • Cancer
  • Ureteral cancer is cancer of the ureters, muscular tubes that propel urine from the kidneys to the urinary bladder. (wikipedia.org)
  • No. 07-6215, Bethesda, MD: National Cancer Institute, pp. 251-262, retrieved 18 October 2013 Transitional Cell Cancer (Kidney/Ureter), National Cancer Institute Urethral Cancer, Department of Urology, University of Miami Leonard M. Miller School of Medicine McCredie M, Stewart JH, Ford JM (1983). (wikipedia.org)
  • gene
  • Human chromosome translocations between the PDGFRB gene and at least any one of 30 genes on other chromosomes lead to myeloid and/or lymphoid neoplasms that are many ways similar to the neoplasm caused by the fusion of the PDGFRA (i.e. platelet derived growth factor receptor A or alpha-type-platelet derived growth factor receptor) gene with the FIP1L1 gene (see FIP1L1-PDGFRA fusion gene. (wikipedia.org)
  • IMPDH2 has been identified as an intracellular target of the natural product sanglifehrin A This gene is up-regulated in some neoplasms, suggesting it may play a role in malignant transformation. (wikipedia.org)
  • Mutations in the gene which codes for PDGFRA, i.e. the PDGFRA gene, are associated with an array of clinically significant neoplasms. (wikipedia.org)
  • mutant
  • the mutant mice exhibit defects in kidney glomeruli because of a lack of mesangial cells but also suffer an ill-defined blood defect characterized by thrombocytopenic, a bleeding tendency, and severe anemia which could be due to blood loss. (wikipedia.org)
  • Neoplasms are mosaics of different mutant cells with both genetic and epigenetic changes that distinguish them from normal cells. (wikipedia.org)
  • While many of the genetic and epigenetic abnormalities in neoplasms are probably neutral evolution, many have been shown to increase the proliferation of the mutant cells, or decrease their rate of death (apoptosis). (wikipedia.org)
  • In this way, a population of mutant cells, called a clone, can expand in the neoplasm. (wikipedia.org)
  • carcinoma in s
  • Transitional cell papilloma, NOS M8120/2 Transitional cell carcinoma in situ Urothelial carcinoma in situ M8120/3 Transitional cell carcinoma, NOS Urothelial carcinoma, NOS Transitional carcinoma M8121/0 Schneiderian papilloma, NOS (C30.0, C31. (wikipedia.org)
  • The pattern of growth of TCCs can be papillary, sessile, or carcinoma-in-situ (CIS). (wikipedia.org)
  • hybridization
  • Diagnosis may include a fluorescence in situ hybridization (FISH) test, computed tomography urography (CTU), magnetic resonance urography (MRU), intravenous pyelography (IVP) x-ray, ureteroscopy, or biopsy. (wikipedia.org)
  • Myeloproliferative
  • Other genetic abnormalities in PDGFRB lead to various forms of potentially malignant bone marrow disorders: small deletions in and chromosome translocations causing fusions between PDGFRB and anyone of at least 30 genes can cause Myeloproliferative neoplasms that commonly involve eosinophilia, eosinophil-induced organ injury, and possible progression to aggressive leukemia (see blow). (wikipedia.org)
  • chronic myelogen
  • Advances in cytogenetics facilitated discovery of chromosome abnormalities in neoplasms, including the Philadelphia chromosome in chronic myelogenous leukemia and translocations in acute myeloblastic leukemia. (wikipedia.org)
  • Cancer
  • Ureteral cancer is cancer of the ureters, muscular tubes that propel urine from the kidneys to the urinary bladder. (wikipedia.org)
  • No. 07-6215, Bethesda, MD: National Cancer Institute, pp. 251-262, retrieved 18 October 2013 Transitional Cell Cancer (Kidney/Ureter), National Cancer Institute Urethral Cancer, Department of Urology, University of Miami Leonard M. Miller School of Medicine McCredie M, Stewart JH, Ford JM (1983). (wikipedia.org)
  • gene
  • IMPDH2 has been identified as an intracellular target of the natural product sanglifehrin A This gene is up-regulated in some neoplasms, suggesting it may play a role in malignant transformation. (wikipedia.org)
  • Human chromosome translocations between the PDGFRB gene and at least any one of 30 genes on other chromosomes lead to myeloid and/or lymphoid neoplasms that are many ways similar to the neoplasm caused by the fusion of the PDGFRA (i.e. platelet derived growth factor receptor A or alpha-type-platelet derived growth factor receptor) gene with the FIP1L1 gene (see FIP1L1-PDGFRA fusion gene. (wikipedia.org)
  • Mutations in the gene which codes for PDGFRA, i.e. the PDGFRA gene, are associated with an array of clinically significant neoplasms. (wikipedia.org)
  • mutant
  • Neoplasms are mosaics of different mutant cells with both genetic and epigenetic changes that distinguish them from normal cells. (wikipedia.org)
  • While many of the genetic and epigenetic abnormalities in neoplasms are probably neutral evolution, many have been shown to increase the proliferation of the mutant cells, or decrease their rate of death (apoptosis). (wikipedia.org)
  • In this way, a population of mutant cells, called a clone, can expand in the neoplasm. (wikipedia.org)
  • the mutant mice exhibit defects in kidney glomeruli because of a lack of mesangial cells but also suffer an ill-defined blood defect characterized by thrombocytopenic, a bleeding tendency, and severe anemia which could be due to blood loss. (wikipedia.org)