• disease
  • This supports our hypothesis that podocyte-matrix interaction via collagen receptors plays an important part in progression of renal fibrosis in Alport disease. (uzh.ch)
  • Supplementing collagen XVIII-deficient mice with exogenous endostatin did not affect the progression of anti-GBM disease. (world-of-links.com)
  • Cell
  • Collagen XVIII/endostatin deficiency altered matrix redesigning enhanced the inflammatory response and advertised capillary rarefaction and vascular endothelial cell damage but did not impact endothelial proliferation. (world-of-links.com)
  • HSPGs get excited about biologic processes such as for example glomerular purification cell adhesion migration proliferation and differentiation 3 that are mediated with the binding of chemokines cytokines enzymes development factors or various other bioactive substances.6 Collagen XVIII (Col 18) is a HSPG connected with BMs of virtually all epithelia and endothelia. (world-of-links.com)
  • development
  • Large epidemiological studies should prove whether the platelet collagen receptor GPIa-IIa T807 allele Is an Independent risk factor for the development of stroke In younger patients. (ehesp.fr)