Mitochondrial cardiolipin involved in outer-membrane protein biogenesis: implications for Barth syndrome. (1/22)

Barth syndrome: an X-linked cause of fetal cardiomyopathy and stillbirth. (2/22)

Mitochondrial dysfunction and organic aciduria in five patients carrying mutations in the Ras-MAPK pathway. (3/22)

Cardiac and skeletal muscle defects in a mouse model of human Barth syndrome. (4/22)

Characterization of a transgenic short hairpin RNA-induced murine model of Tafazzin deficiency. (5/22)

Barth syndrome mutations that cause tafazzin complex lability. (6/22)

Impaired cardiac reserve and severely diminished skeletal muscle O(2) utilization mediate exercise intolerance in Barth syndrome. (7/22)

Substrate metabolism during basal and hyperinsulinemic conditions in adolescents and young-adults with Barth syndrome. (8/22)

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