TY - JOUR. T1 - Detection of an atypical 7q11.23 deletion in Williams syndrome patients which does not include the STX1A and FZD3 genes. AU - Botta, A.. AU - Novelli, G.. AU - Mari, A.. AU - Novelli, A.. AU - Sabani, M.. AU - Korenberg, J.. AU - Osborne, L. R.. AU - Digilio, M. C.. AU - Giannotti, A.. AU - Dallapiccola, B.. PY - 1999. Y1 - 1999. N2 - We present two patients with the full Williams syndrome (WS) phenotype carrying a smaller deletion than typically observed. The deleted region spans from the elastin gene to marker D7S1870. This observation narrows the minimal region of deletion in WS and suggests that the syntaxin 1A and frizzled genes are not responsible for the major features of this developmental disorder and provides important insight into understanding the genotype-phenotype correlation in WS.. AB - We present two patients with the full Williams syndrome (WS) phenotype carrying a smaller deletion than typically observed. The deleted region spans from the elastin gene to marker ...
ndividuals with Williams syndrome typically show relatively poor visuospatial abilities in comparison to stronger verbal skills. However, individuals level of performance is not consistent across all visuospatial tasks. The studies assessing visuospatial functioning in Williams syndrome are critically reviewed, to provide a clear pattern of the relative difficulty of these tasks. This prompts a possible explanation of the variability in performance seen, which focuses on the processing demands of some of these tasks. Individuals with Williams syndrome show an atypical processing style on tests of construction, which does not affect tests of perception.. ...
Williams syndrome is characterized by a wide spectrum of symptoms and physical features that vary greatly in range and severity, even among affected family members. Individuals with Williams syndrome will not have all the symptoms listed below. Some affected individuals do not have heart (cardiac) abnormalities; others may not have elevated levels of calcium in the body (hypercalcemia). In addition, the severity of these symptoms often varies greatly from case to case.. Some children with Williams syndrome may have a low birth weight, feed poorly, and fail to gain weight and grow at the expected rate (failure to thrive). Symptoms such as vomiting, gagging, diarrhea, and constipation are common during infancy. Some affected infants may have elevated levels of calcium in their blood (hypercalcemia), leading to loss of appetite, irritability, confusion, weakness, easy fatigability, and/or abdominal and muscle pain. Calcium levels usually return to normal around the age of 12 months. However, in ...
Most individuals with Williams syndrome have an affinity to music. They are touched by music in ways not usually seen in the general population. It is quite common for those with Williams syndrome to be reduced to tears by classical music, disturbed by music played in minor chords or moved to dance and laugh by playful and happy music.
The Williams-Beuren syndrome (WBS) is a sporadic congenital disorder characterized by a multisystem developmental impairment. This syndrome is caused by a microdeletion in chromosome 7q11.23 that encompasses loss of the elastin locus.. Elastin, which is part of the extracellular matrix, controls proliferation of vascular smooth muscle cells (VSMCs) and stabilizes arterial structure. Loss of elastin gene in WBS patients has been claimed to provide a biological basis for the abnormal elastic fibre properties leading to cardiovascular abnormalities like supravalvular aortic stenosis (SVAS), hypertension, arteriosclerosis and stenosis in more than 50% of WBS children.. These cardiovascular pathologies result in important consequences and neither curative nor preventive medicinal treatments exist at this time. Surgery is needed in more than half cases, while it is often leading to complications.. Minoxidil is a well-known antihypertensive drug used in adults and children. Furthermore, according to ...
Williams syndrome (WS) is a developmental disorder that affects many parts of the body. Facial features frequently include a broad forehead, short nose, and full cheeks, an appearance that has been described as "elfin". Mild to moderate intellectual disability with particular problems with visual spatial tasks such as drawing and fewer problems with language are typical. Those affected often have an outgoing personality and interact readily with strangers. Problems with teeth, heart problems, especially supravalvular aortic stenosis, and periods of high blood calcium are common. Williams syndrome is caused by a genetic abnormality, specifically a deletion of about 27 genes from the long arm of one of the two chromosome 7s. Typically this occurs as a random event during the formation of the egg or sperm from which a person develops. In a small number of cases it is inherited from an affected parent in an autosomal dominant manner. The different characteristic features have been linked to the loss ...
Married to my college sweetheart Shawn for 14 years, we have six children - Jamie (11), Clare (9), Simon (7), Violet (5), Eliza (3), and Cecily (1). Our second child, Clare, was born with a rare genetic condition, Williams syndrome, severe congenital heart defects (pulmonary stenosis and aortic stenosis), a Chiari Malformation, mid-aortic syndrome, and kidney issues. She has undergone one open heart surgery and ten cardiac catheterizations to repair her cardiac and vascular defects and has also undergone a frenulectomy (tongue-tie release) and an angio-embolization of her radial artery to repair an A-V fistula and aneurysm. On May 1, she had her longest surgery yet - a bypass graft of her abdominal aorta, a bypass graft of her SMA vessel, and a kidney re-transplant (kidneys transplanted elsewhere in her body). I began this blog when Clare was an infant to chronicle our life on this new journey into Williams syndrome and raising a child with special needs and a chronic heart condition. As our ...
Educators face unique challenges teaching children with Williams syndrome, but with knowledge of the learning styles of students with WS, your students can experience success in the classroom. For a one page fact sheet on the educational profile for sudents with Williams syndrome, click here.Connecting the ws cognitive profile to educational strategiesWith school age children we see:
Williams Syndrome (WS) is a rare genetic disorder characterized by mild to moderate mental retardation or learning difficulties, a distinctive facial appearance, and a unique personality that combines over-friendliness and high levels of empathy with
Fairy tales tell of wee folk who spend their lives singing and dancing. A rare genetic disorder called Williams Syndrome lends scientific support to the legends. Drummer Jeremy Vest is among those who are diagnosed with Williams Syndrome.
A hallmark feature of Williams-Beuren Syndrome (WBS) is a generalized arteriopathy due to elastin deficiency, presenting as stenoses of medium and large arteries and leading to hypertension and other cardiovascular complications. Deletion of a functi
Author Summary A fundamental question in current biomedical research is to establish a link between genomic variation and phenotypic differences, which encompasses both the seemingly neutral diversity, as well as the pathological variation that causes or predisposes to disease. Once the primary genetic cause(s) of a disease or phenotype has been identified, we need to understand the biochemical consequences of such variants that eventually lead to increased disease risk. Such phenotypic effects of genetic differences are supposedly brought about by changes in expression levels, either of the genes affected by the genetic change or indirectly through position effects. Thus, transcriptome analyses seem appropriate proxies to study the consequences of structural variation, such as the 7q11.23 deletion present in individuals with Williams-Beuren syndrome (WBS). Here, we present an approach that takes experimental data into account instead of relying solely on functional annotation, following the rationale
Q: Is there really a condition that makes kids be born looking like elves?A: There is a congenital syndrome called Williams-Beuren Syndrome (WBS, sometimes just called Williams syndrome) that manifests with facial features of a broad forehead, a small upturned nose, full lips with a long upper lip, a small chin, a starburst pattern in the colored parts of their eyes (more pronounced in blue or green-eyed patients) and full cheeks, as well as an outgoing, sociable personality.
Q: Is there really a condition that makes kids be born looking like elves?A: There is a congenital syndrome called Williams-Beuren Syndrome (WBS, sometimes just called Williams syndrome) that manifests with facial features of a broad forehead, a small upturned nose, full lips with a long upper lip, a small chin, a starburst pattern in the colored parts of their eyes (more pronounced in blue or green-eyed patients) and full cheeks, as well as an outgoing, sociable personality.
Q: Is there really a condition that makes kids be born looking like elves?A: There is a congenital syndrome called Williams-Beuren Syndrome (WBS, sometimes just called Williams syndrome) that manifests with facial features of a broad forehead, a small upturned nose, full lips with a long upper lip, a small chin, a starburst pattern in the colored parts of their eyes (more pronounced in blue or green-eyed patients) and full cheeks, as well as an outgoing, sociable personality.
Q: Is there really a condition that makes kids be born looking like elves?A: There is a congenital syndrome called Williams-Beuren Syndrome (WBS, sometimes just called Williams syndrome) that manifests with facial features of a broad forehead, a small upturned nose, full lips with a long upper lip, a small chin, a starburst pattern in the colored parts of their eyes (more pronounced in blue or green-eyed patients) and full cheeks, as well as an outgoing, sociable personality.
Q: Is there really a condition that makes kids be born looking like elves?A: There is a congenital syndrome called Williams-Beuren Syndrome (WBS, sometimes just called Williams syndrome) that manifests with facial features of a broad forehead, a small upturned nose, full lips with a long upper lip, a small chin, a starburst pattern in the colored parts of their eyes (more pronounced in blue or green-eyed patients) and full cheeks, as well as an outgoing, sociable personality.
La síndrome de Williams-Beuren és una malaltia del neurodesenvolupament causada per una deleció comú dentre 26 i 28 gens contigus a la regió 7q11.23, dificultant lestabliment de relacions genotip-fenotip. Lús de models de ratolí pot augmentar el coneixement sobre la malaltia, el paper dels gens delecionats, les vies moleculars afectades i els futurs tractaments. En aquesta tesi shan usat diversos models de ratolí, les seves cèl·lules i teixits per tal de descriure i definir fenotips, gens i vies moleculars desregulades i per descobrir elements modificadors i nous tractaments. Per últim, sha definit un nou motiu dunió per Gtf2i, uns dels gens delecionats que codifica per un factor de transcripció amb un rol central en la síndrome, proporcionats possible nous gens diana de vies moleculars desregulades. Els resultats obtinguts revelen el paper essencial dels models de ratolí per a lestudi de la síndrome de Williams-Beuren, proporcionen noves opcions terapèutiques i ...
MS Thesis: EXPRESSION AND FUNCTION OF WILLIAMS SYNDROME TRANSCRIPTION FACTOR (WSTF) IN THE NEURAL DEVELOPMENT OF XENOPUS LAEVIS Imitation Switch (ISWI) is a member of the SWI2/SNF2 superfamily of ATP-dependent chromatin remodelers. Twenty different ISWI complexes have been identified so far in yeast, Drosophila, Xenopus and mammals. Three ISWI-containing complexes, WICH, ACF and CHRAC, have been characterized in Xenopus. Loss of ISWI function in Xenopus embryos results in severe defects in neural and eye development, including loss of retinal differentiation and formation of cataracts. We have begun to dissect the contributions of individual ISWI-dependent complexes to development, by using in situ hybridization and antisense morpholino knockdowns against subunits unique to different ISWI-containing complexes. Here I have investigated the WICH complex in Xenopus and have targeted the WSTF subunit. Whole mount in situ hybridization shows WSTF localized in the neural tissue including eye, brain, ...
Spoken language is probably the most important form of social interaction between people and, maybe not surprisingly, we found that the way the brain processes language mirrors the contrasting social phenotypes of Williams syndrome and autism spectrum disorders," says lead author Inna Fishman, Ph.D., a neuropsychologist in the Laboratory of Cognitive Neuroscience at the Salk, who conceived the study together with Debra Mills, Ph.D., currently a reader at Bangor University in UK. Autism spectrum disorders and Williams syndrome are both neurodevelopmental disorders but their manifestations couldnt be more different: While autistic individuals live in a world where objects make much more sense than people do, people with Williams syndrome are social butterflies who bask in other peoples attention. Despite myriad health problems, generally low IQs and severe spatial problems, people with Williams syndrome are irresistibly drawn to strangers, look intently at peoples faces, remember names and ...
About a year ago, if you remember, Self, we were just coming out of kidney failure. Williams was pretty new to us. I was overwhelmed all of the time. I turned to that woman, and I just told her what she had wanted to know. I told her about having a new baby, who was born sick, and how long it was before I could hold him. We were living away from our family, and then we took our four month old and moved to Finland for a semester abroad. I told her how my husband spent all day in school and how I was in a concrete, unfurnished apartment sitting on a sleeping bag with a baby who screamed all day and all night, and clawed at his face until he bled because something was wrong and we didnt know what. I told her that as soon as we hit America we took him to a doctor where they did test after test after test, and finally it was determined that his severe heart problems, among other things, pointed toward a strange thing called WILLIAMS SYNDROME. And how after even more testing, it was determined that ...
About a year ago, if you remember, Self, we were just coming out of kidney failure. Williams was pretty new to us. I was overwhelmed all of the time. I turned to that woman, and I just told her what she had wanted to know. I told her about having a new baby, who was born sick, and how long it was before I could hold him. We were living away from our family, and then we took our four month old and moved to Finland for a semester abroad. I told her how my husband spent all day in school and how I was in a concrete, unfurnished apartment sitting on a sleeping bag with a baby who screamed all day and all night, and clawed at his face until he bled because something was wrong and we didnt know what. I told her that as soon as we hit America we took him to a doctor where they did test after test after test, and finally it was determined that his severe heart problems, among other things, pointed toward a strange thing called WILLIAMS SYNDROME. And how after even more testing, it was determined that ...
Author(s): Hanson, Kari | Advisor(s): Semendeferi, Katerina | Abstract: The evolution of the human brain has yielded advanced cognitive capacities supporting the development of language, technologically advanced material culture, and highly complex social behavior that has allowed for the development of the rich diversity of human cultures. Comparative neuroanatomy in evolutionary perspective continues to make great strides in characterizing and defining unique elements of the human neuroanatomical phenotype at the gross and microscopic level that underlie these key behavioral adaptations. In conjunction with these studies, an understanding of the functional implications of derived anatomical traits is gained through analyses of neurodevelopmental disorders, which help to define a spectrum of variation in the diversity of human brain phenotypes. Williams syndrome (WS) is a rare neurodevelopmental disorder caused by a hemideletion of ~1.6 Mb (25-28 genes) on human chromosome 7q11.23, a highly dynamic
Sakurai, T., Dorr, N. P., Takahashi, N., McInnes, L. A., Elder, G. A. and Buxbaum, J. D. (2011), Haploinsufficiency of Gtf2i, a gene deleted in Williams Syndrome, leads to increases in social interactions. Autism Res, 4: 28-39. doi: 10.1002/aur.169 ...
The molecular and neural mechanisms regulating human social-emotional behaviors are fundamentally important but largely unknown; unraveling these requires a genetic systems neuroscience analysis of human models. Williams Syndrome (WS), a condition caused by deletion of ∼28 genes, is associated with a gregarious personality, strong drive to approach strangers, difficult peer interactions, and attraction to music. WS provides a unique opportunity to identify endogenous human gene-behavior mechanisms. Social neuropeptides including oxytocin (OT) and arginine vasopressin (AVP) regulate reproductive and social behaviors in mammals, and we reasoned that these might mediate the features of WS. Here we established blood levels of OT and AVP in WS and controls at baseline, and at multiple timepoints following a positive emotional intervention (music), and a negative physical stressor (cold). We also related these levels to standardized indices of social behavior. Results revealed significantly higher median
... (WS) is the rare neurodevelopmental disorder characterized by: an elfin or distinctive facial appearance, along with a low nasal bridge; a demeanor which is unusually cheerful, and ease with strangers; strong language skills with developmental delay. This is the forum for discussing anything related to this health condition
Williams syndrome is a rare neurodevelopmental disorder caused by a deletion of about 26 genes from the long arm of chromosome 7. It is characterized by mental disability, heart defects and unusual facial features.
Does any one know the icd-9-cm code for Williams Syndrome? I have looked through the book and cannot find this syndrome under anything.
Riby, Deborah and Doherty-Sneddon, Gwyneth (2009) Eye movements and gaze interpretation by individuals with Williams syndrome and autism. In: Society for Research in Child Development (SRCD) biennial meeting, 2nd April 2009, Denver, Colorado. Full text not available from this repository. (Request a copy ...
View Notes - Notes Day 2 from PSYCH 100 at UMass (Amherst). Genes and Personality: Williams Syndrome (Video) 1 in 25,000 children are born with it. They are characteristically affectionate and
David Dobbs has an interesting article in The New York Times Magazine about Williams syndrome; a disorder characterized by verbosity and hypersociality in concert with abstraction capacities so attenuated that most suffers are mentally retarded. The piece juggles many phenomena, from general to domain specific intelligences and the interaction between environment and genetic biases which shape the minds developmental arc ...
There is no known cure for Williams syndrome as of 2015, but social training, physical therapy, speech therapy, monitoring of blood and heart vessel defects and occupational therapy are some of the...
This category is for all associations or organizations that promote education, research, and advocacy for patients with Williams Syndrome, their families and caregivers.
Meet Chloe, the 11-year-old whose powerful smile is bringing together supporters and scientists to advance research on Williams syndrome.
Williams syndrome associated Celiac Disease information including symptoms, diagnosis, misdiagnosis, treatment, causes, patient stories, videos, forums, prevention, and prognosis.
My name is Kelsey Braaten and I work with Sam and Dustin Devary. I wanted to help … Kelsey Rae Braaten needs your support for Westons Williams Syndrome story
Williams Syndome (WS) is as complex neurodevelopmental disorder characterized by vascular and heart disease, mental retardation, characteristic facial features, and chararteristic personality. WS, which is usually sporadic, affects approximately 1 in 20,000 live births and is caused by a particular deletion of about 1.5 Mb of chromosome 7q11.23. My lab has shed light on the anatomical and cognitive basis for WS. While WS patients have cognitive gifts, such as notable linguistic abilities, they also have deficits, such as visuospatial deficits. WS patients also show significant anatomical differences, with a reduces perimeter of the corpus collosum and amygdalar nuclei. The major current goal in the field is to connect the genetic basis to these anatomical and cognitive differences. About 28 genes are deleted in WS; four such genes are Elastin, Lim kinase, WSTF and GTF21. An elastin deletion results in supravalcular aortic sclerosis (SVAS), which is commin in WS patients. Lim kinase absence affects actin
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Further parsing the consequences of GTF2IRD1 versus GTF2I on spatial structure and social behavior once was hampered by the small number of cases with fewer than the most common gene deletions and limited cognitive data, clarifies Korenberg. To distinguish the roles of both genes, postdoctoral research and researcher first author Li Dai, Ph.D., combed the genomes of 17 Williams Syndrome patients to recognize those who had lost only 1 GTF2I gene. This allowed identification of a girl who got retained GTF2I but didnt match the classical explanation of the disorder. Getting this girl was extremely exciting, Korenberg stated. Her case had so very much power to explain the part of these genes. When the Salk researchers tested the girl to measure her IQ and social behavior, they found her scores in vocabulary, information digesting, comprehension, arithmetic, and the ability to surface finish partially finished drawings to be considerably closer to normal than most sufferers with Williams syndrome. ...
USC defensive line Leonard Williams admitted Saturday that it is difficult to avoid thinking about whether to turn pro early. It is understandable when virtually every mock draft lists Williams as a top-five pick.. "I think about it all the time," Williams said. "Everyones asking about it. Its going to be the hardest decision I will probably ever make.. "As cheesy as it may be, its going to come down to how I feel in my heart.". ESPN draft guru Mel Kiper lists Williams as the No. 2 prospect behind Oregon quarterback Marcus Mariota. Williams, who was disappointed to be a second-team Walter Camp All-American, was chosen a first-team All-American by ESPN.com.. He is the first defensive end since Marlin McKeever to earn first-team All-American twice. Of course it should be noted that Williams also plays inside even though he is listed as a defensive end.. USC coach Steve Sarkisian reminisced about facing Williams when Washington played USC in 2012.. "He was a little bit of a novelty (as a ...
When its merger with Energy Transfer Equity was on the brink of collapse, Williams warned investors that it would need to cut its dividend by a material amount if the deal did not close. That is just what happened, with the company announcing a 68.8% reduction shortly after the deal imploded. However, the method of the reduction was surprisingly smart, with Williams choosing to reinvest the excess cash to support its MLP Williams Partners (NYSE:WPZ) by acquiring additional units in a distribution reinvestment program (DRIP). Doing so would enable Williams Partners to maintain its lucrative payout because its parent would provide it with some of the cash it needed to fund growth capex. If everything goes according to plan, both Williams and Williams Partners expect to be in the position to start growing their respective payouts in 2018. That has the potential to drive accelerated income growth at Williams, which by that time will own a much larger stake in its MLP. Further, this agreement will ...
Last night Taylor and I ditched the kids and went to Williams schools charity silent auction. While we didnt win anything, we did have a good time. We had a chance to talk to Williams teacher too which was nice. For me there is always a moment of awkwardness approaching her. Other than Taylor and I, she spends the most time with William but we arent friends. I dont hang out with her, I dont know a lot about her life etc. So really we only have William in common and I never know what to say to her exactly. Obviously I want to hear about William in school but I also want to be polite and not bombard her with questions about my son. Anyway, the awkwardness wore off pretty quickly. We had a great conversation (about William) and I learned that hes doing advanced work in math. We already knew that he could count beyond 100. (Though he forgets 13, 14, and 15 EVERY TIME he counts, but he remembers 113, 114, and 115. Weird, right?) Apparently she has introduced William to some of the older kids ...
After ONeill, Williams is perhaps the best dramatist the United States has yet produced. Born in his grandfathers rectory in Columbus, Mississippi, Williams and his family later moved to St. Louis. There Williams endured many bad years caused by the abuse of his father and his own anguish over his introverted sister, who was later permanently institutionalized. Williams attended the University of Missouri, and, after time out to clerk for a shoe company and for his own mental breakdown, also attended Washington University of St. Louis and the University of Iowa, from which he graduated in 1938. Williams began to write plays in 1935. During 1943 he spent six months as a contract screenwriter for MGM but produced only one script, The Gentleman Caller. When MGM rejected it, Williams turned it into his first major success, The Glass Menagerie (1945). In this intensely autobiographical play, Williams dramatizes the story of Amanda, who dreams of restoring her lost past by finding a gentleman caller ...
Mary Elizabeth Williams is an American writer and commentator. She is a staff writer for the online magazine Salon. She has also written for The New York Times, The Nation, and other publications. As a commentator, she has made appearances on MSNBC, Today, and NBC Nightly News. In 2009, Williams released a memoir titled Gimme Shelter. Mary Elizabeth Williams grew up in Jersey City, New Jersey. She has described herself as a practicing Catholic. In August 2010, Williams was diagnosed with malignant melanoma and underwent surgery. In August 2011, she was rediagnosed with stage IV melanoma. Later that year, she entered a stage I clinical trial for an experimental immunotherapy cancer drug, with which she had some success. Williams has documented her experiences with cancer on Salon. Larry Smith (March 9, 2009). "Interview: Mary Elizabeth Williams, Author of Gimme Shelter". Smith Magazine. Retrieved 11 May 2012. Mary Elizabeth Williams. "The story so far.." MaryElizabethWilliams.net. Retrieved 11 ...
A new study finds that adults with Williams syndrome-who are extremely social and trusting-use Facebook and other social networking sites frequently and are especially vulnerable to online victimization.
On March 31, 2014, Williams was announced as a new coach for the seventh season of The Voice, replacing CeeLo Green.[59] On May 18, 2015, Team Pharrell had 16-year-old Sawyer Fredericks win the eighth season of The Voice. In June 2014, it was announced that Williams would make a guest appearance on the docu-series Sisterhood of Hip Hop.[60] Williams was the executive producer of Atlanta rapper T.I.s ninth studio album, Paperwork, which was released on October 21, 2014 by Grand Hustle and Columbia Records.[61] In May 2014, Williams curated an art show named after his album, "Girl," at the Galerie Perrotin in Paris, France. The show included 37 artists including Takashi Murakami, JR, Daniel Arsham, and Marina Abramovic among others."[62] Comme des Garçons developed a unisex fragrance with Williams[63] scheduled for release in late 2014. Kaws designed the bottle artwork.[64]. In January 2015, Williams and Al Gore announced that they are teaming up to create a 7-continent "Live Earth" concert on ...
Partner: Theory and Multi-disciplinary Practice. Vollman, A., Anderson, E., and McFarlane J. (eds), 4 th edition, Chapter 6, pp. 80-91. London: Lipincott, Williams and Wilkins.. Williams, L. (2011) Developing Personal Skills: Building Capacity for Individual, Collective and Socio-political Empowerment. In Canadian Community as Partner: Theory and Multi-disciplinary Practice. Vollman, A., Anderson, E., and McFarlane, J. (eds), 3rd edition, pp. 87-102. London: Lipincott, Williams and Wilkins.. Williams, L. and Mumtaz, Z. (2008) Being alive well? Power-knowledge as a countervailing force to the realization of mental well-being for Canadas Aboriginal youth. International Journal of Mental Health Promotion, 10(4): 21-31. http://dx.doi.org/10.1080/14623730.2008.9721773. Williams, L. (2008) Developing Personal Skills: Empowerment. In Canadian Community as Partner: Theory and Practice. Vollman, A., Anderson, E., and McFarlane, J. (eds), 2nd edition, pp. 94-112. London: Lipincott, Williams and ...
The following is a guest post from Doug Williams, who writes from the federal prison camp at Florence, Colorado. Williams, who for decades had taught people how to pass or beat polygraph "tests" without incident (and whose website, Polygraph.com, remains online), was targeted for entrapment in a federal criminal investigation dubbed Operation Lie Busters. On 21 February 2013, federal agents searched Williams home and office, seizing business records. Williams was later indicted on two counts of mail fraud and three counts of witness tampering, and pled guilty during a trial held in May 2015. On 30 October 2015, Williams began serving a two-year prison sentence. The only crimes of which Williams was convicted are those that federal agents conceived and stage managed.. It is now very obvious that my book From Cop to Crusader: The Story of My Fight Against the Dangerous Myth of Lie Detection has been distributed to just about every polygraph operator on the face of the earth. And while it is true ...
Gospel Music Legend Melvin Williams Receives First EMMY Award Nomination. New Documentary on PBS Follows Williams Journey from Mississippi Cotton Fields through Five-Decade Career in Music and Entertainment (New York, NY - May 24, 2018) - Gospel Music legend Melvin Williams, a member of the award-winning Williams Brothers, has received a 2018 Southeast EMMY Award nomination for Special Event Coverage of his new documentary, "Melvin Williams: Down Home Gospel." The film, which examines Williams journey from his roots in Smithdale, Mississippi, to a stellar, five-decade career in music and entertainment, will premiere nationally on PBS during Gospel Heritage Month 2018 (September) in more than 200 markets. The film initially aired on Mississippi Public Broadcasting on March 4, 2017, and January 26, 2018.. The 44th Annual Southeast Emmy Awards will be held on Saturday, June 16, 2018, at the Grand Hyatt Buckhead in Atlanta, Georgia. Hosted by CNNs Anchor Fredricka Whitfield, the Southeast EMMY ...
Ian Williams practiced Wednesday for the first time since he suffered a broken left ankle last season.. Williams return comes at a time when the 49ers are in need of a replacement for veteran nose tackle Glenn Dorsey, who suffered a torn biceps earlier in camp and is sidelined indefinitely.. "Our last look at Ian was last camp and into the second game ," 49ers defensive coordinator Vic Fangio said.. Hence, Fangio and others in the 49ers organization are eager to see how well Williams has recovered from an injury that required four surgeries.. Williams participated in the teams morning walk-through Wednesday. He is slated to take part in the full-scale practice this afternoon.. At that point, Williams said, he should get a better feel for where hes at in his near-year-long recovery.. "Its different running around, cutting, everything like that; thats the easy part," Williams said. "Anybody can do combine drills like that. But when youre pushing against somebody thats 350 pounds, thats ...