Catecholaminergic polymorphic ventricular tachycardia is characterized by polymorphic ventricular tachycardia in the structurally normal heart. It is typically triggered by physical activity, emotional stress or catecholamine infusion. Ventricular tachycardia can lead to dizziness, syncope, seizures, ventricular fibrillation and sudden death.. The Catecholaminergic polymorphic ventricular tachycardia NGS panel consists of nine genes: ANK2, CALM1, CALM2, CALM3, CASQ2, KCNJ2, RYR2, TECRL and TRDN.. Copy number variation (CNV) analysis of the catecholaminergic polymorphic ventricular tachycardia genes is also offered as a panel. Additionally, CTGT offers a comprehensive test (both NGS and CNV panels) for these genes. Panel genes are also offered as individual sequencing and deletion/duplication tests unless otherwise indicated.. ...
Objectives. The purpose of this study was to determine the predictors of electrically induced ventricular tachycardia in a large sample of patients with unexplained syncope and to examine the value of the signal-averaged electrocardiogram (ECG) in those patient subsets with varying pretest probability of ventricular tachycardia.. Background. In patients with unexplained syncope, electrophysiologic study can provide important diagnostic information, such as inducibility of ventricular tachycardia. The signal-averaged ECG can predict inducible ventricular tachycardia, but its utility has not been prospectively studied in a large group of patients with unexplained syncope.. Methods. At six hospitals, 189 consecutive patients with unexplained syncope underwent signal-averaged ECG and electrophysiologic studies.. Results. Ventricular tachycardia was induced in 28 patients (15%). Univariate predictors of ventricular tachycardia included history of previous myocardial infarction, reduced left ...
The selective infusion of ethanol into the coronary circulation supplying the site of origin of incessant ventricular tachycardia has been demonstrated to abolish this arrhythmia in selected patients. The present study was designed to evaluate the efficacy and safety of the intracoronary ethanol ablation technique in patients with paroxysmal ventricular tachycardia related to prior myocardial infarction. Twenty-three patients with sustained monomorphic ventricular tachycardia that was refractory to conventional antiarrhythmic drug therapy were prospectively studied. After induction of ventricular tachycardia by programmed electrical stimulation, the response of the arrhythmia to the infusion of radiographic contrast medium or saline solution into the ostia of the native coronary arteries and coronary artery bypass grafts was assessed. If ventricular tachycardia was reliably interrupted by injections into the proximal coronary artery or bypass graft, the vessel was cannulated with a steerable guide wire
Paroxysmal ventricular tachycardia is most often associated with organic heart disease and in this setting is of ominous prognostic import (1-3). Less commonly, this arrhythmia occurs in young individuals without underlying heart disease; in these patients the disorder is generally considered benign (4). However, in some of these patients the episodes of paroxysmal ventricular tachycardia may lead to syncope and sudden death (2, 5). Recently, the efficacy of the beta-adrenergic blocking drugs in the treatment of tachyarrhythmias has been recognized (6, 7), and these drugs have been employed with success in certain patients with recurrent ventricular tachycardia (8-11). Electrical pacemakers ...
A new operative technique of sequential map-guided subendocardial resection (SER) was used in 45 consecutive patients for the treatment of sustained ventricular tachycardia due to coronary artery disease. This technique is characterized by map-guided SER or cryothermic ablation during normothermic cardiopulmonary bypass, followed by repeated sequences of programmed stimulation to assess adequacy of resection. The patients mean age was 59 +/- 10 years and the mean left ventricular ejection fraction was 34 +/- 12%. Twenty-five (56%) patients had a history of myocardial infarction within the previous 2 months. After ventriculotomy, 34 patients (76%) had inducible monomorphic ventricular tachycardia. These patients underwent repeated sequences of ventricular tachycardia induction and mapping during normothermic bypass followed by successive SER or cryothermic ablation until sustained monomorphic ventricular tachycardia was no longer inducible. Twenty-seven patients had a total of 60 discrete, ...
The morphology of the tachycardia depends on its cause and the origin of the re-entry electrical circuit in the heart. In monomorphic ventricular tachycardia, the shape of each heart beat on the ECG looks the same because the impulse is either being generated from increased automaticity of a single point in either the left or the right ventricle, or due to a reentry circuit within the ventricle. The most common cause of monomorphic ventricular tachycardia is scarring of the heart muscle from a previous myocardial infarction (heart attack). This scar cannot conduct electrical activity, so there is a potential circuit around the scar that results in the tachycardia. This is similar to the re-entrant circuits that are the cause of atrial flutter and the re-entrant forms of supraventricular tachycardia. Other rarer congenital causes of monomorphic VT include right ventricular dysplasia, and right and left ventricular outflow tract VT. Polymorphic ventricular tachycardia, on the other hand, is most ...
The morphology of the tachycardia depends on its cause and the origin of the re-entry electrical circuit in the heart. In monomorphic ventricular tachycardia, the shape of each heart beat on the ECG looks the same because the impulse is either being generated from increased automaticity of a single point in either the left or the right ventricle, or due to a reentry circuit within the ventricle. The most common cause of monomorphic ventricular tachycardia is scarring of the heart muscle from a previous myocardial infarction (heart attack). This scar cannot conduct electrical activity, so there is a potential circuit around the scar that results in the tachycardia. This is similar to the re-entrant circuits that are the cause of atrial flutter and the re-entrant forms of supraventricular tachycardia. Other rarer congenital causes of monomorphic VT include right ventricular dysplasia, and right and left ventricular outflow tract VT. Polymorphic ventricular tachycardia, on the other hand, is most ...
The use of implantable cardioverter-defibrillators (ICD) in patients with reduced ejection fraction after myocardial infarction (MI) has been shown to reduce cardiovascular death and total mortality (1). However, defibrillator shocks for recurrent ventricular tachycardia (VT) can be painful and anxiety provoking for patients. Antiarrhythmic therapy can be used to prevent recurrent episodes of VT. However, such therapy may be incomplete and is often associated with adverse effects. Catheter ablation has the potential to prevent recurrent VT without the need for long-term antiarrhythmic therapy. According to the American Heart Association consensus statement, catheter ablation is recommended "for symptomatic sustained monomorphic VT, including VT terminated by an ICD, that recurs despite antiarrhythmic drug therapy or when antiarrhythmic drugs are not tolerated or not desired" (2).. The mechanism of VT after MI in the vast majority of patients is re-entry, which is facilitated by slow conduction ...
What is ventricular tachycardia? Ventricular tachycardia is an arrhythmia represented by rapid heartbeats that originate in the ventricles. During ventricular tachycardia the heart may beat inefficiently and as a result the blood pressure may decrease very much. This type of tachycardia may lead to life-threatening arrhythmias and cardiac arrest. Treatment options for ventricular tachycardias include medications, catheter ablation or placement of an implantable cardioverter-defibrillator (ICD).
The purpose of this study is to determine if early ablation (i.e., ablation of ventricular tachycardia in patients with infrequent VT episodes) is more effective than medical therapy alone for the treatment of ischemic ventricular tachycardia in patients with Implantable Cardioverter Defibrillators (ICDs) who continue to have episodes of ventricular tachycardia despite drug therapy ...
A closed-heart method for treating ventricular tachycardia in a myocardial infarct patient afflicted with ventricular tachycardia is disclosed. The method comprises, first, defining a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) of the patient, and then ablating the thin layer of spared myocardial tissue by a closed-heart procedure with an ablation catheter. Apparatus for carrying out the method is also disclosed. Also disclosed is a method for prognosing the likelihood of ventricular tachycardia occuring in a myocardial infarct patient not previously diagnosed as afflicted with ventricular tachycardia. The method comprises detecting a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) in the patient.
A closed-heart method for treating ventricular tachycardia in a myocardial infarct patient afflicted with ventricular tachycardia is disclosed. The method comprises, first, defining a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) of the patient, and then ablating the thin layer of spared myocardial tissue by a closed-heart procedure with an ablation catheter. Apparatus for carrying out the method is also disclosed, Also disclosed is a method for prognosing the likelihood of ventricular tachycardia occuring in a myocardial infarct patient not previously diagnosed as afflicted with ventricular tachycardia. The method comprises detecting a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) in the patient.
This meta-analysis is limited by the studies included in the analysis. All of the studies were observational studies, and therefore they are susceptible to selection bias. There were significant differences in the patients included and the strategy used when performing VT ablations. Although the overall strategy was to target all inducible VTs, only 1 study indicated ablation of all clinically relevant VTs as a predetermined end point.23 Table II in the Data Supplement indicates the specific protocols and catheters used in the analyzed studies.. Another important limitation is the definition of the clinical VT. The definition used in the selected studies is not uniform and is not based on the 12-lead ECG in all studies. Because 12-lead ECGs are not available for all patients, VT electrogram morphology from implantable cardioverter defibrillators can be used instead with similar accuracy and have been found to be helpful in identifying the clinical VT.5 This definition was used by only one, but ...
Idiopathic ventricular tachycardia (VT) has been described from the epicardial surface of the left ventricle (LV), usually near the summit of the LV in the outflow tract. Ablation of these VTs may be possible by delivery of energy within the coronary venous system or directly on the epicardial surface. We describe a distinct syndrome of focal epicardial VT induced by catecholamine infusion that arises from the crux of the heart. Among 340 cases of idiopathic VT referred for catheter ablation, 4 patients were identified with a clinical syndrome of catecholamine sensitive VT that was mapped to the epicardial surface at the crux of the heart. There were 3 males and 1 female (age 31-79 yrs, mean 58). VT was sustained in all patients and associated with syncope or near syncope in 3 of 4 pts. The LVEF was ,0.55 in 3 pts and mildly depressed (0.45) in 1 pt. In all pts VT could be induced with programmed stimulation or burst pacing from the right ventricular apex but required the infusion of ...
TY - JOUR. T1 - Mode of induction of ventricular tachycardia and prognosis in patients with coronary disease. T2 - The multicenter unsustained tachycardia trial (MUSTT). AU - Piccini, Jonathan P.. AU - Hafley, Gail E.. AU - Lee, Kerry L.. AU - Fisher, John D.. AU - Josephson, Mark E.. AU - Prystowsky, Eric N.. AU - Buxton, Alfred E.. PY - 2009/8/1. Y1 - 2009/8/1. N2 - Mode of Induction in MUSTT. Introduction: Programmed stimulation is an important prognostic tool in the evaluation of patients with an ejection fraction ≤40% after myocardial infarction. Many believe that ventricular tachycardia (VT) requiring 3 ventricular extrastimuli (VES) for induction is less likely to occur spontaneously and has less predictive value. However, it is unknown whether the mode of VT induction is associated with long-term prognosis. Methods and Results: We analyzed a cohort of 371 patients enrolled in MUSTT who had inducible monomorphic VT and who were not treated with antiarrhythmic drugs or an implantable ...
Ventricular tachycardia is one of the commonest cause of sudden death in chronic chagas disease. As most ventricular tachycardias originate from scar in patients with heart disease, catheter ablation is an important step in patient treatment. Identification of fibrosis prior to ablation of sustained ventricular tachycardia (SVT) might reduce the time of anesthesia, procedure time, radiation exposure and possibly the risk of complications. Knowledge of arrhythmia circuit within scar allows planning strategies for each procedure. Condreanu et al. stablished that voltages inferior to 6.52 mV (unipolar) and 1.54mV (bipolar) are useful tools in detecting scar during electroanatomic mapping. Accuracy, however when compared to magnetic resonance imaging is limited due to difficulties in maintaining good contact between ablation catheter and ventricular wall. Contact force catheters might help increase accuracy of voltage mapping because they allow detection of poor contact areas. Although the threshold ...
TY - JOUR. T1 - Calmodulin mutations causing catecholaminergic polymorphic ventricular tachycardia confer opposing functional and biophysical molecular changes. AU - Søndergaard, Mads T. AU - Sorensen, Anders B. AU - Skov, Louise L. AU - Kjaer-Sorensen, Kasper. AU - Bauer, Mikael C. AU - Nyegaard, Mette. AU - Linse, Sara. AU - Oxvig, Claus. AU - Overgaard, Michael Toft. N1 - This article is protected by copyright. All rights reserved.. PY - 2015/1/14. Y1 - 2015/1/14. U2 - 10.1111/febs.13184. DO - 10.1111/febs.13184. M3 - Journal article. C2 - 25557436. VL - 282. SP - 803. EP - 816. JO - F E B S Journal. JF - F E B S Journal. SN - 1742-464X. IS - 4. ER - ...
The following pages link to Catecholaminergic Polymorphic Ventricular Tachycardia: View (previous 50 , next 50) (20 , 50 , 100 , 250 , 500) ...
Ventricular tachycardia is a very rare fetal arrhythmia accounting for fewer than 2% of fetal tachycardias. We describe a fetus presenting at 30 weeks gestation with ventricular tachycardia at a rate of 220 beats per min and fetal hydrops. The tachycardia was unresponsive to flecainide but was co...
A case of a 51 year old patient with a history of myocardial infarction (MI) and recurrent ventricular tachycardia (VT) is presented. Three months after MI the patient underwent coronary angioplasty and one year later received prophylactic implantable cardioverter-defibrillator (ICD) due to complex ventricular arrhythmias, detected on Holter ECG monitoring, and depressed left ventricular ejection fraction. Later on the patient started to experience palpitations and ICD shocks during physical activity (cycling). Interrogation of the ICD memory showed appropriate shocks due to slow (160 betas/min) VT. The device was reprogrammed and new antitachycardia pacing (ATP) algorithms were enabled, however, it occurred proarrhythmic due to the ATP-induced acceleration of VT rate. Finally, in April 2005 he received 37 appropriate ICD shocks during a few hours. The patient was selected for RF ablation and underwent successful procedure with the use of the electro-anatomical CARTO mapping system ...
An accelerating ventricular tachycardia ending in fibrillation can be initiated in isolated hearts by procaine injections after the administration of epinephrine, but not in untreated hearts. The run of tachycardia consists of a supraventricular or idioventricular impulse followed by an accelerating train of premature systoles coupled to it. Fibrillation results when the train reaches a frequency of 10 to 12 per second. In hearts with their bundles of His cut, epinephrine injections alone cause only a ventricular tachycardia of constant frequency. Therefore, the initiation of ventricular arrhythmias by procaine must entail the production of A-V blockade and some process which causes the tachycardia to accelerate.. ...
We describe a scar-related reentrant ventricular tachycardia circuit with a proximal segment in an endocardial basal septal scar and an exit in a region of slow conduction in a non-overlapping region of epicardial basal lateral scar. The 12-lead EKG
The objective was to determine the characteristics of heart rate variability and ventricular arrhythmias prior to the onset of ventricular tachycardia (VT) in patients with an implantable cardioverter defibrillator (ICD). Sixty-eight beat-to-beat time series from 13 patients with an ICD were analyzed to quantify heart rate variability and ventricular arrhythmias. The episodes of VT were classified in one of two groups depending on whether the sinus rate in the 1 min preceding the VT was greater or less than 90 beats per minute. In a subset of patients, increased heart rate and reduced heart rate variability was often observed up to 20 min prior to the VT. There was a non-significant trend to higher incidence of premature ventricular complexes (PVCs) before VT compared to control recordings. The patterns of the ventricular arrhythmias were highly heterogeneous among different patients and even within the same patient. Analysis of the changes of heart rate and heart rate variability may have predictive
We report a case of a patient with idiopathic dilated cardiomyopathy and recurrent ventricular tachycardias refractory to antiarrhythmic treatment with amiodarone. A cardioverter defibrillator implantation was performed by the transvenous technique,
Srivatsa UN, Danielsen B, Anderson I, Amsterdam E, Pezeshkian N, Yang Y, White RH. Risk predictors of stroke and mortality after ablation for atrial fibrillation: the California experience 2005-2009. Heart Rhythm. 2014 Nov;11(11):1898-903. Epub 2014 Jul 15.. Miller JM, Pezeshkian NG, Yadav AV. Catheter mapping and ablation of right ventricular outflow tract ventricular tachycardia. J Cardiovasc Electrophysiol. 2006 Jul;17(7):800-2.. ...
en] Catecholaminergic polymorphic ventricular tachycardia is important to be diagnosed as an underlying disease in children with syncope and normal heart, because of its poor prognosis. CASE REPORT: A 3-year-old boy was referred for stress and emotion induced syncope. Primary ventricular arrhythmia, consisting of salvos of bidirectional ventricular tachycardia, was reproducibly induced by physical exertion. The syncopal events and severe arrhythmia disappeared with beta-blocking therapy. CONCLUSION: Despite its rare occurrence, catecholaminergic polymorphic ventricular tachycardia is an important cause of stress and emotion induced syncope and sudden death in children ...
An implantable cardioverter capable of delivering pacing and cardioversion level therapies. The device distinguishes between stable and unstable ventricular tachyarrhythmias by monitoring the progression of atrial cycle lengths during the detected ventricular tachyarrhythmia. A detected increase in atrial cycle lengths during the early stages of the detected ventricular tachycardia is taken as an indication of hemodynamically unstable ventricular tachycardia.
BVT is an infrequent arrhythmia that has nevertheless mesmerized electrophysiologists for many years. It is most commonly observed under conditions of digitalis intoxication and in advanced heart disease.22 On ECG, BVT is manifested as an alternation in the polarity of the QRS axis in some of the leads; the remaining leads may demonstrate changes in morphology.22 The tachycardia is often regular, occurs in brief salvoes, and often resolves spontaneously or may degenerate into PVT or VF. The alternating pattern is usually associated with bundle branch block morphology in the precordial leads, with the alternating QRS complexes differing from each other in amplitude and duration. Since its first description in 1922,23 several hypotheses have been postulated for the mechanism of BVT, including enhanced automaticity with the existence of 2 separate ventricular foci22,24 or even reentry.22 More recently, the demonstration of RyR2 gain-of-function mutations in patients with familial CPVT has led to ...
Learn more about Ventricular Tachycardia at Reston Hospital Center DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...
Pulseless ventricular tachycardia is a temporary but life-threatening condition, where the heart moves rapidly but ineffectively, causing severe risk if not treated.
Pulseless ventricular tachycardia is a temporary but deadly condition, where the heart moves rapidly but ineffectively, causing severe risk if not treated.
We present a case of Great Cardiac Vein cryoablation in order to suppress idiopathic epicardial Ventricular Tachycardia after failed Radiofrequency ablation via the epicardium and GCV.
The ICD aborts sudden cardiac death in patients with symptomatic ventricular tachyarrhythmias. The role of this device in preventing sudden death in patients with asymptomatic tachyarrhythmias is less clear. The landmark MADIT study found that ICD was an effective therapy for a select group of patients with asymptomatic unsustained ventricular tachycardia at very high risk for sudden death. Patients in MADIT had previous MIs, mean ejection fractions of 25% to 27%, and electrophysiologically inducible sustained tachyarrhythmias that could not be suppressed with procainamide. These patients had a very high risk for sudden death and may represent the group that is least likely to respond to antiarrhythmic therapy (1). Despite very low ejection fractions, only 50% to 60% were treated with angiotensin-converting enzyme inhibitors. More optimal treatment of systolic dysfunction could decrease sudden death and overall mortality and lower the absolute benefits of ICD therapy. Whether participants ...
Simulation-driven engineering has put rockets in space, airplanes in the sky, and self-driving cars on the road. Computational approaches have also contributed to advancements in clinical medicine and human health.1-3 In the arena of cardiac care, the recent emphasis on personalized medicine has provided a significant impetus for the development of predictive approaches combining imaging and computational modeling that can be applied to the diagnosis and treatment of heart rhythm disorders. A major advance in this direction is the creation and translation into clinical practice of novel imaging- and simulation-based strategies for predicting an individuals risk of sudden cardiac death (SCD) and for the noninvasive planning of optimal personalized antiarrhythmia therapies. Clinical decisions about the stratification of patients for SCD risk resulting from arrhythmia and for determining the optimal targets for antiarrhythmia ablation therapies could greatly benefit from such targeted developments ...
The following case is being reported because of the huge and unprecedented amount of medications required to control successfully a desperate cardiac complication, i.e., paroxysmal ventricular tachycardia. It was fairly clear from the experience during one of the episodes in this case that the tachycardia of itself might prove fatal. This permitted us-and encouraged us-to use the massive amounts of medication that proved to be necessary to control a subsequent attack. ...
An dysrhythmia of the heart is an irregular heartbeat rhythm. Ventricular tachycardia is an dysrhythmia in which the lower chambers of the heart, the ventricles, beat unusually fast.. The heart rate is measured with an electrocardiogram, or ECG. This is a machine that measures the electrical impulses from the patients heart. This is displayed on a monitor or ECG graph paper. The boxes on the graph paper measure time. Five small boxes are equal to one large box. The large box represents two-tenths of a second.. The atrial contraction is represented by the P wave. This is an upward, or positive wave of the line on the graph. The ventricular contraction is displayed by the QRS complex. The QRS complex is composed of three waves, the Q, R, and S waves. The Q wave is the first downward, or negative movement on the graph. The R wave is the positive movement following the Q wave. It rises above the starting point of the Q wave. The S wave is a negative movement following the R wave. The S wave returns ...
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Catecholaminergic polymorphic ventricular tachycardia (CPVT) is a potentially fatal cardiac arrhythmia in individuals with a structurally normal heart. The disorder is characterized by syncope, typically beginning in the first decade of life, which may be triggered by physical activity or intense emotion. In patients with CPVT, stress- induced release of catecholamines causes a dysfunction of calcium-ion channel in myocytes. The ion channel dysfunction induces ventricular arrhythmias, which can lead to syncope or sudden cardiac death. Spontaneous recovery from the arrhythmia is possible, but the ventricular tachycardia can progress to ventricular fibrillation and sudden death. The incidence of CPVT within the population is not precisely known, but is estimated to be 1:10,000. Symptoms include syncope, dizziness, arrhythmia, and sudden cardiac death. Diagnosis may prove difficult, due to normal echocardiogram and electrocardiogram at a resting state. Testing must be performed under ...
Ventricular tachycardia is a fast, abnormal heart rate that begins in the lower chambers of the heart. It can become life-threatening if it lasts more than a few seconds.
The main objective of this clinical case presentation was to found the real cause of dangerous arrhythmias (frequents premature ventricular bates polymorphic, polytope and repeated passes of none sustained ventricular tachycardia) at a young patient. I present the clinical case of a young woman patient 21 years old, who came at the consultation for irregular palpitations after increase physical effort-mountain climbing-her favorite sport activity. At the objective examination were found irregular heart bates HR=98bates/min, without murmur heart or added sounds, BP=150/90mmHg, normal vesicular breath sound on the lung. The EKG showed: sinus rhythm, HR=98bates/minutes, many premature ventricular bates looks polymorphic, polytope and a short pass of a non sustained ventricular tachycardia (less than 30 seconds). The patient was monitored and followed antiarrhythmic therapy with xilin iv bolus 1mg/kg body and after that double dose of xilin 1% i.v. in perfusion during the first 24 hours with ...
RESULTS: We identified 3 trials of 1-2 years duration. These trials included a total of 573 patients who received fish oil and 575 patients who received a control. Meta-analysis of data collected at 1 year showed no overall effect of fish oil on the relative risk of implantable cardioverter defibrillator discharge. There was significant heterogeneity between trials. The second largest study showed a significant benefit of fish oil (relative risk [RR] 0.74, 95% confidence interval [CI] 0.56-0.98). The smallest showed an adverse tendency at 1 year (RR 1.23, 95% CI 0.92-1.65) and significantly worse outcome at 2 years among patients with ventricular tachycardia at study entry (log rank p = 0.007 ...
Regarding MTWA and ICDs, the stake was drilled in the heart of that debate at the American Heart Assoc meeting 2007 when the MASTER II trial results were presented. Recall that the aim of the MASTER study was to determine whether an abnormal T-wave alternans test result is associated with subsequent life-threatening ventricular tachyarrhythmias in patients for whom an ICD was indicated on MADIT II criteria. The results? The mean age of patients was 65 years, mean ejection fraction at baseline was 24% and most patients had a prior history of heart failure. Indeterminate tests were reported in 107 patients, of whom 69 were retested with a definitive result obtained in 59%. Baseline MTWA test was negative in 214 patients and "non-negative" in 361 patients. A primary endpoint event occurred in 22 MTWA negative patients (10%) and 48 "non-negative" patients (13%), with no difference between the groups (HR 1.26 CI 0.76-2.09, p=0.37). Subgroup analysis suggested that patients with a QRS interval ,120 ...
Accurate guidance of an ablation catheter is critical in the RF ablation (RFA) of ventricular tachycardia (VT). With current technologies, it is challenging to rapidly and accurately localize the site of origin of an arrhythmia, often restricting treatment to patients with hemodynamically stable arrhythmias. We investigated the effectiveness of a new guidance method, the inverse solution guidance algorithm (ISGA), which is based on a single-equivalent dipole representation of cardiac electrical activity and is suitable for patients with hemodynamically unstable VT. Imaging was performed in homogeneous and inhomogeneous saline-filled torso phantoms in which a catheter tip was guided toward a stationary electrical dipole source over distances of more than 5 cm. Using ISGA, the moving catheter tip was guided to within 0.61 plusmn0.43 and 0.55 plusmn0.39 mm of the stationary source in the homogeneous and inhomogeneous phantoms, respectively. This accuracy was achieved with less than ten movements of ...
MHPs/participants emphasized the importance in providing an empathetic presence while also coupon for cialis by manufacturer engaging in spiritual networking. The mortality rate among young women (ages 20-44) began to increase during the early 1990s.. Role of sympathovagal balance in the initiation of idiopathic ventricular tachycardia originating from right ventricular outflow tract. Clinical and radiological aspects of biliary cost of tadalafil generic at walgreens ileus (10 personal cases) Mechanical property characterization of prostate tumors can enhance the results obtained by palpation by providing quantitative and precise diagnostic information to compare cialis vs viagra vs levitra surgeons. The effect of different fiber concentrations on the surface roughness of provisional crown and fixed partial denture resin. Eight children with chronic SMA were tested, in addition to 9 obligate heterozygote carriers of the gene.. However, their interactions with soil microorganisms are still poorly ...
Question posted in: tachyarrhythmia, parkinsons disease - Additional details: Last year I was diagnosed with SVT, which I received a catheter ...
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I have been very active (running, soccer, weightlifting) and have been healthy (heart rate 60 or less; blood press 112/78). Over the past several months I had a steady decline in stamina. Tried to run ...
I have been in nursing for 33 years and have worked in a variety of settings including hospice, long term care, med-surg, supervision, cath lab, ER, special procedures and critical care. I enjoy working as a float nurse because it gives me a variety of clinical experiences. I am also a CPR, ACLS, and PALS instructor at our local hospital.. ...
Participants went from experiencing 6,577 episodes in the 3 months before treatment to just four during 46 patient-months after the blanking period.
The patient recovered quickly and his headache and nausea disappeared. A 66-year-old woman was admitted buy lipitor online to our hospital because of left ventricular failure and nonsustained ventricular tachycardia. GnRH activates ERK1/2 leading to alfuzosin buy online the induction of ...