Tachycardia, also called tachyarrhythmia, is a heart rate that exceeds the normal resting rate. In general, a resting heart rate over 100 beats per minute is accepted as tachycardia in adults. Heart rates above the resting rate may be normal (such as with exercise) or abnormal (such as with electrical problems within the heart). The upper threshold of a normal human resting heart rate is based on age. Cutoff values for tachycardia in different age groups are fairly well standardized; typical cutoffs are listed below: 1-2 days: Tachycardia > 159 beats per minute (bpm) 3-6 days: Tachycardia >166 bpm 1-3 weeks: Tachycardia >182 bpm 1-2 months: Tachycardia >179 bpm 3-5 months: Tachycardia >186 bpm 6-11 months: Tachycardia >169 bpm 1-2 years: Tachycardia >151 bpm 3-4 years: Tachycardia >137 bpm 5-7 years: Tachycardia >133 bpm 8-11 years: Tachycardia >130 bpm 12-15 years: Tachycardia >119 bpm >15 years - adult: Tachycardia >100 bpm Heart rate is considered in the context of the prevailing clinical ...

Mechanistic studies of human heart failure are complicated by the prolonged time course of development of the disease, the technical challenges of isolating cardiac tissue or cells from explanted hearts, the inability to investigate the early time course of cellular alterations, and the lack of control over experimental conditions. Thus, it is fortunate that the canine tachycardia-induced heart failure model so closely reproduces the known hemodynamic and ionic changes that have been identified in human hearts. The present findings indicate that, in addition to the electrophysiological changes noted in earlier studies, significant alterations in Ca2+ handling occur in isolated myocytes from failing hearts, following the general pattern of human studies. Through biochemical and functional measurements in the same hearts, we have found strong evidence in support of the hypothesis that the induction of heart failure triggers a shift in the balance of cytosolic Ca2+ extrusion mechanisms from SR Ca2+ ...

Successful Ablation for Atrial Tachycardia Originated from Sinus Venosa with Tachycardia-Induced Cardiomyopathy. . Biblioteca virtual para leer y descargar libros, documentos, trabajos y tesis universitarias en PDF. Material universiario, documentación y tareas realizadas por universitarios en nuestra biblioteca. Para descargar gratis y para leer online.

Fetal Tachycardia (FT) is described as increase in baseline fetal heart rate (FHR) above 160bpm. Mild fetal tachycardia is described as 161-180bpm and severe tachycardia is defined as greater than 180bpm for at least three minutes. The fetal tachycardia causes include maternal fever, dehydration or anxiety, maternal ketosis, medications like anticholinergic medications, sympathomimetic medications like terbutaline, fetal movement, preterm fetus, maternal thyrotoxicosis and maternal anaemia1. Fetal tachycardia is considered significant (any range ,160-180bpm) in the presence of maternal pyrexia as Chorioamnionitis is suspected. Fetal arrhythmia or congenital defect is associated with FHR more than 200 bpm. Baseline FHR tachycardia represents an increase in sympathetic and or a decrease in parasympathetic autonomic nervous system tone1.. Complicated fetal tachycardia in the presence of decelerations or maternal fever qualify the decision for delivering the baby in view of fetal distress and ...

Tachycardia refers to a condition of an irregularly rapid beating of the heart. Tachycardia is defined as a heart rate greater than 100 beats per minute, while a normal heart beats is measured at 72 beats per minute. Tachycardia occurs when the electrical impulses that coordinate heartbeats do not function properly and cause the heart to beat irregularly fast. In some situations, an increased heart rate can be normal, such as during exercise and stress. However, depending on the extent of the Tachycardia and the overall health of the patient, tachycardia may be dangerous and require serious medical attention.. If the heart rate is abnormally increased for an extended period of time, oxygen and carbon dioxide levels in the bloodstream can be dramatically altered. Tachycardia can cause stress on the heart because the heart muscle is forced to take less time to relax between each contraction. Patients with Tachycardia may often feel out of breath because the faster the heart beats, the more oxygen ...

TY - JOUR. T1 - Tachycardia in Post-Infarction Hearts. T2 - Insights from 3D Image-Based Ventricular Models. AU - Arevalo, Hermenegild. AU - Plank, Gernot. AU - Helm, Patrick. AU - Halperin, Henry R. AU - Trayanova, Natalia. PY - 2013/7/2. Y1 - 2013/7/2. N2 - Ventricular tachycardia, a life-threatening regular and repetitive fast heart rhythm, frequently occurs in the setting of myocardial infarction. Recently, the peri-infarct zones surrounding the necrotic scar (termed gray zones) have been shown to correlate with ventricular tachycardia inducibility. However, it remains unknown how the latter is determined by gray zone distribution and size. The goal of this study is to examine how tachycardia circuits are maintained in the infarcted heart and to explore the relationship between the tachycardia organizing centers and the infarct gray zone size and degree of heterogeneity. To achieve the goals of the study, we employ a sophisticated high-resolution electrophysiological model of the infarcted ...

A pacing system provided with a mode switching feature and ventricular rate regularization (VRR) function adapted to stabilize or regularize ventricular heart rate during chronic or paroxysmal atrial tachyarrhythmia. In a preferred embodiment, the pacing system nominally operates in an atrial synchronized pacing mode such as DDD or DDDR pacing mode. In response to detection of atrial rhythm characteristics consistent with an atrial tachyarrhythmia, e.g., atrial fibrillation, a mode switch into a non-atrial synchronized, ventricular rate regularization pacing mode, e.g. DDIR or VDIR pacing mode, is made. If the VRR function is programmed on, the ventricular pacing rate based upon a rate responsive sensor derived ventricular pacing rate modulated on a beat by beat basis by preceding intrinsic or paced ventricular events, the stability of the intrinsic ventricular heart rate, and any atrial pace events to regularize the ventricular pacing rate. The pacing system may also be permanently programmed to the

The freeMD virtual doctor has found 32 conditions that can cause Abdominal Tenderness and Tachycardia. There are 6 common conditions that can cause Abdominal Tenderness and Tachycardia. There are 4 somewhat common conditions that can cause Abdominal Tenderness and Tachycardia. There are 7 uncommon conditions that can cause Abdominal Tenderness and Tachycardia. There are 15 rare conditions that can cause Abdominal Tenderness and Tachycardia.

This page includes the following topics and synonyms: Tachycardia due to Medications, Drug Induced Tachycardia, Toxin Induced Tachycardia.

The study of conversion of AF to sinus rhythm provides information on changes associated with the resumption of atrial contraction and AV synchrony leading to a regularized, rate-controlled ventricular response. Previous studies have documented improvement in cardiac output ([75]) and cardiopulmonary exercise testing variables of ventricular rate control, maximal oxygen uptake and anaerobic threshold ([76, 77]) after cardioversion to sinus rhythm. More recently, a small, prospective study of patients with a diagnosis of idiopathic dilated cardiomyopathy and chronic AF reported a significant increase in left ventricular ejection fraction after pharmacologic or electrical cardioversion to sinus rhythm ([78]). Whether improvement in cardiac function was due to restoration of atrial systolic function and AV synchrony versus reversal of an underlying cardiomyopathy associated with rapid and irregular ventricular rates during AF was not resolved by this study. Subsequent study of atrial and ...

The best 16 synonyms for tachycardia, including: hypotension, tachypnoea, dysrhythmias, bronchospasm, palpitation, dyspnoea, stridor, low blood pressure, hypoxaemia, ventricular-fibrillation, oedema and more... Find another word for tachycardia at YourDictionary.

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The rhythm is a regular WCT ( = Wide-Complex Tachycardia) at ~ 210/minute without sign of atrial activity. Dr. Smith has reviewed the principal differential diagnosis. I dont think QRS duration is particularly helpful here, given that the patient is a 12-year old child (for whom normal QRS duration is a bit less than in adults). So the QRS is definitely wide, though not excessively so - but probably overlaps most entities to be considered. What strikes me - is the obvious suggestion of bifascicular block (RBBB/LAHB), though of atypical morphology. In an otherwise healthy child (who does not have congenital heart disease or other structural heart abnormality) - one would expect a more typical RBBB morphology with a clear triphasic appearance with taller right-rabbit ear in lead V1, instead of the atypical picture we see here in V1,V2,V3. Considering this and integrating it with the important Learning Points Dr. Smith emphasizes (ie, that fascicular VT often occurs in otherwise normal hearts) - ...

Most of us (assuming no pediatric cardiologists are listening) do not see a large number of pediatric arrhythmias (certainly far less than weve all seen in adults). As a result - level of comfort in both diagnosis and management tends to be less than that attained with adult arrhythmias. As wonderfully emphasized by Dr. Smith by the 2 pediatric arrhythmia examples shown here - accurate determination of rate is CRITICAL in the assessment of pediatric arrhythmias. For this determination - I have found use of the Every-other-Beat (or sometimes, Every-third-Beat) method a user-friendly way to rapidly and accurate determine heart rate for any regular fast rhythm. While most of us recall, 300- 150 - 100 - 75 - 60 - 50 as the answers for rate calculation when the R-R interval of a regular tachycardia is 1, 2, 3, 4, 5 or 6 large boxes, respectively - this becomes problematic with very fast rates. Thats because small differences in measurements may translate into significant under- or ...

Improved methods and devices perform tachycardia detection and anti-tachycardia pacing (ATP) to convert a tachycardia (e.g., VT or AT) to normal sinus rhythm. According to one embodiment, an anti-tachycardia pacing method includes sensing, during sinus rhythm, first and second cardiac signals at first and second sites, respectively, in a patients heart. The first and second sites include left and right ventricles or left and right atria, for example. The method further includes sensing third and fourth cardiac signals at the first and second sites, respectively, during a tachycardia (e.g., ventricular tachycardia or atrial tachycardia). The cardiac signals are processed to provide respective values. One or more anti-tachycardia pacing pulses are delivered at the site closest to the reentrant circuit based on a comparison of a first ratio of the first and third values and a second ratio of the second and fourth values. Unipolar sensing of the cardiac signals may be employed by, for example,

Tachycardia: Abnormally rapid heartbeat, usually with a HEART RATE above 100 beats per minute for adults. Tachycardia accompanied by disturbance in the cardiac depolarization (cardiac arrhythmia) is called tachyarrhythmia.

TITLE PAGETitle: Interesting changeover from short VA to long VA tachycardia with single atrial premature depolarisation: What is the mechanism?Short title: Concomitant AVNRT and atrial tachycardia: Killing two birds with one stone.

What is tachycardia arrhythmia? Learn about tachycardia arrhythmia (fast heartbeat), including causes, risk factors, symptoms, diagnosis and treatment from the cardiology experts at Mercy Health.

Fascinating case with a number of interesting insights to learn. Diagnosis of alternate preexcitation on the last tracing is subtle, but important to recognize. The 2 other thoughts I considered in my differential diagnosis for this last ECG were: i) electrical alternans; and ii) ventricular bigeminy with fusion from end-diastolic PVCs - but neither fit (thats because we see DELTA waves in several leads on this tracing that provide the diagnosis). Otherwise - the T wave peaking we see on the tachycardia ECGs is extremely interesting. I have often seen T wave peaking with tachycardia (and on stress tests) in otherwise healthy young adults - but I dont expect ST elevation. In addition, this patient was apparently markedly dehydrated on each occasion when the ECG was done. Hyperkalemia (perhaps secondary to volume contraction) not uncommonly leads to T wave peaking with recurrent episodes, but again supposedly not associated with ST elevation (Wonder what serum K+ values were in this patient ...

Tachycardia refers to a fast resting heart rate, usually over 100 beats per minute. Tachycardia can be dangerous, depending on its underlying cause and on

I. Problem/Condition. Tachycardias include all heart rhythms with a rate , 100 bpm. They can be divided into two primary categories: ) Narrow Complex Tachycardias (NCT) which have a QRS duration , 120 msec ) Wide Complex Tachycardias (WCT) which have a QRS duration , 120 msec The NCTs are due to rapid activation of…. ...

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BACKGROUND: Catheter ablation (CA) therapy is the first choice treatment in adults with heart rhythm disturbances. The arrhythmias in adults are mainly conditioned by coronary disease. Etiology of arrhythmias in children is mostly associated with inherited heart disorders. According to the current guidelines the CA is widely used in children, indicating the need to make it more achievable in pediatric population. AIM: To assess efficacy and safety of CA in children with different types of arrhythmias on the initial learning curve at newly built Ablation Center in The Independent Pediatric Hospital of Medical University of Warsaw ...

Most arrhythmia including Tachycardia are not serious However, any abnormality associated with the heart can quickly develop into a serious, or even life

An interesting technicality in the use of the PERC rule to rule out pulmonary embolism is the tachycardia component - it asks not whether the patient is tachycardic at the time of the application of the rule, or whether tachycardia was sustained throughout the emergency department stay, but instead whether the patient had (as described by Jeff Kline in his great review article on PE diagnosis and risk stratification): 3. Pulse ,100 beats/min during entire stay in ED. Meaning, even transient tachycardia may suggest a life-threatening diagnosis, even if it resolves while the patient is in the emergency department, and were probably PERCing out a whole bunch of patients inappropriately, at least according to Kline (who, notably, testifies a whole bunch as an expert witness in cases of missed pulmonary emboli).. I recently had a handful of patients in whom concerning blood pressures were measured and documented, which then resolved when vital signs were re-checked or after a small quantity of ...

You are working one evening in the emergency department when a 60-something year old female is slotted for a room. Her chief complaint? Fever, weakness, vomiting. Seeing that her triage heart rate was 157, you leave your granola bar where it is and immediately walk into the room to assess her. You see an elderly-appearing female in moderate respiratory distress. Her temperature is 38.2, blood pressure is 125/87, RR is 32, oxygen saturation is 93% on 5L NC. She has a history of a bone-marrow transplant and is chronically immunosuppressed. She endorses poor PO intake and several episodes of emesis over the last few days. She says that she came in today when she developed some shortness of breath as well. She denies any chest pain or palpitations. On exam, her mucous membranes are dry, her abdomen non-tender, and her breath sounds are decreased in the right base. You are a bit disturbed by the looks of her rhythm strip on the monitor so you get a 12-lead EKG ...

A Computational Study of the Effects of Tachycardia-Induced Remodeling on Calcium Wave Propagation in Rabbit Atrial Myocytes: In atrial cardiomyocytes without a

Learn more about Tachycardia at Reston Hospital Center DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...

Learn more about Tachycardia at Grand Strand Medical Center DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...

Tachycardia is a condition where a persons heart is beating abnormally fast. When we become more active our heart rate increases to meet the increased

Atrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics (costs for program #250319) ✔ University Hospital Halle (Saale) ✔ Department of Cardiology and Angiology (Department of Internal Medicine III) ✔ BookingHealth.com

Atrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics (costs for program #144277) ✔ Academic Hospital Neuperlach ✔ Department of Cardiology, Pneumology and Internal Intensive Care ✔ BookingHealth.com

Tachycardia is a rapid heart rate caused by a problem in the hearts electrical system. Find out more about this common heart rhythm disorder (arrhythmia).

A pacemaker-mediated tachycardia (PMT) can be defined as any condition in which a pacemaker paces the ventricles at rates that are inappropriately fast. This can be due to (1) a rate response setting that is too sensitive, (2) tracking of atrial noise (such as what may occur with electromagnetic interference), (3) inappropriate pacemaker mani...

A heart rate of more than 100 beats per minute (BPM) in adults is called tachycardia. Whats too fast for you may depend on your age and physical condition.

Rapid and abnormal increase in heart rate more than 100 beats a minute in rest is tachycardia. It is caused by abnormal electrical impulses originated in the heart chambers with a physiological or pathological background.

Its been a while since I been on here, but I must say life is good, I am truly blessed. My pvcs are almost completely gone, I may feel only a couple a week and I havent had a tachycardia attack in over three months, I must say again God is good. I was once afraid to live on my own because if my heart issues, but I recently moved into my own house three weeks ago. I posted a while back about the doctor putting me on Mirtazapine for anxiety and weight gain, it has really helped me alot, I started off at 111lbs, I am now 134lbs ...

Question - Suffering from SVT tachycardia. Prescribed with metroprolol. Is it safe to go for skiing?. Ask a Doctor about diagnosis, treatment and medication for Increased heart rate, Ask a Cardiologist

how high can an axiety attact drive ones HR up? to 110, 120, 130?? I fhigh, one will confuse the adrenaline caused high HR that stops in a few minutes with tachycardia. More and more I believe, aft...

i did a post on here regarding tachycardia warnings well this is part 2 after suffering complications in hospital which resulted in very low potassium it would seem that i am back to square one...

Dont just change employers, change your life with Healthworks, Inc. This webinar focuses on the core concepts of a basic electrophysiology study and tools to induce tachycardia.

A disruption in the normal electrical impulses controlling the pumping action of the heart causes tachycardia, according to Mayo Clinic. Many different factors cause these disruptions and the related...

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Had cathetor ablation following a 189 heart beat which I could not control (prior to that over a number of years had, say 120 beats - I could stop by holding my breath a few times and it would disappe

I have been experiencing a fast, pounding heart rate for over a month now. It gets worse when I stand up. I get light headed, very short of breath, dizzy, weak, and feel as if Im going to faint. I wen...

I have been wearing a chest hr monitor for the last week and have been concerned that I seem to be tachycardiac in the afternoons. My resting heart...

DI-fusion, le Dépôt institutionnel numérique de lULB, est loutil de référencementde la production scientifique de lULB.Linterface de recherche DI-fusion permet de consulter les publications des chercheurs de lULB et les thèses qui y ont été défendues.

TY - JOUR. T1 - Chronic orthostatic intolerance. T2 - A disorder with discordant cardiac and vascular sympathetic control. AU - Furlan, Raffaello. AU - Jacob, Giris. AU - Snell, Marie. AU - Robertson, David. AU - Porta, Alberto. AU - Harris, Paul. AU - Mosqueda-Garcia, Rogelio. PY - 1998/11/17. Y1 - 1998/11/17. N2 - Background - Chronic orthostatic intolerance (COI) is a debilitating autonomic condition in young adults. Its neurohumoral and hemodynamic profiles suggest possible alterations of postural sympathetic function and of baroreflex control of heart rate (HR). Methods and Results - In 16 COI patients and 16 healthy volunteers, intra-arterial blood pressure (BP), ECG, central venous pressure (CVP), and muscle sympathetic nerve activity (MSNA) were recorded at rest and during 75°tilt. Spectral analysis of RR interval and systolic arterial pressure (SAP) variabilities provided indices of sympathovagal modulation of the sinoatrial node (ratio of low-frequency to high-frequency components, ...

Objectives. The purpose of this study was to determine the predictors of electrically induced ventricular tachycardia in a large sample of patients with unexplained syncope and to examine the value of the signal-averaged electrocardiogram (ECG) in those patient subsets with varying pretest probability of ventricular tachycardia.. Background. In patients with unexplained syncope, electrophysiologic study can provide important diagnostic information, such as inducibility of ventricular tachycardia. The signal-averaged ECG can predict inducible ventricular tachycardia, but its utility has not been prospectively studied in a large group of patients with unexplained syncope.. Methods. At six hospitals, 189 consecutive patients with unexplained syncope underwent signal-averaged ECG and electrophysiologic studies.. Results. Ventricular tachycardia was induced in 28 patients (15%). Univariate predictors of ventricular tachycardia included history of previous myocardial infarction, reduced left ...

There are several ways to classify ventricular tachycardia, such as morphology or based on the duration of the episodes. From a morphological point of view, ventricular tachycardia can be divided into monomorphic ventricular tachycardia, scar related monomorphic ventricular tachycardia, polymorphic ventricular tachycardia and RVOT tachycardia. The second type of tachycardia mentioned here is the most common, especially with patients who have already suffered from a heart attack and it can also lead to death. Based on the duration, ventricular tachycardia can be divided into two types: non-sustained ventricular tachycardia if the fast rhythm ends itself within thirty seconds and sustained ventricular tachycardia if the rhythm lasts longer than thirty seconds.. Determining what causes is can be very complicated and requires the input of a specialized doctor and several tests, the most common of which is effectuated with the help of an EKG. The manifestations of ventricular tachycardia can be very ...

This condition can cause dizziness when moving to an upright position and can be triggered by infections. They found that older people, women, and those who had five or more symptoms in the first week of becoming ill with Covid-19 were more likely to develop long Covid. Researchers have analysed data from the COVID Symptom Study app to discover who is most at risk of developing long Covid. The BHF is supporting the following research projects: The BHF is supporting the following research projects: The BHF is supporting the following research projects: The most common symptoms are feeling lightheaded, palpitations (being aware of your heartbeat) and fatigue. The most common symptoms are feeling lightheaded, palpitations (being aware of your heartbeat) and fatigue. There is emerging evidence that a few people who develop long Covid have similar symptoms to people with a condition known as postural tachycardia syndrome, or postural orthostatic tachycardia syndrome (PoTS).. ...

The Postural Tachycardia Syndrome (POTS) is the most common disorder seen in autonomic clinics. Cardinal hemodynamic feature of this chronic and debilitating disorder of orthostatic tolerance is an exaggerated orthostatic tachycardia (≥30 bpm increase in HR with standing) in the absence of orthostatic hypotension. There are multiple pathophysiological mechanisms that underlie POTS. Some patients with POTS have evidence of elevated sympathoneural tone. This hyperadrenergic state is likely a driver of the excessive orthostatic tachycardia. Another common pathophysiological mechanism in POTS is a hypovolemic state. Many POTS patients with a hypovolemic state have been found to have a perturbed renin-angiotensin-aldosterone profile. These include inappropriately low plasma renin activity and aldosterone levels with resultant inadequate renal sodium retention. Some POTS patients have also been found to have elevated plasma angiotensin II (Ang-II) levels, with some studies suggesting problems with

Radiofrequency ablation of ventricular tachycardia is now well established in the management of patients with focal ventricular tachycardia occurring without structural heart disease.1 ,2 ,11 Although high success rates have been reported in this context and much has been learnt about mapping and ablation, they form a small proportion of the total population of patients with ventricular tachycardia.8 ,9 Ischaemic heart disease is the dominant cause of ventricular tachycardia, and RF ablation in this setting is beset with problems. The traditional approach has been to induce ventricular tachycardia and then map the induced rhythm and perform ablation during the tachycardia, seeking to terminate it.. The conventional approach to mapping arrhythmias has been to identify the earliest ventricular activation, which is assumed to be at, or close, to the ventricular tachycardia origin. This approach works well in focal ventricular tachycardia or at surgery where a large endocardial resection is ...

Wide QRS tachycardia may be due to ventricular tachycardia (VT), supraventricular tachycardia (SVT) with aberrant conduction, or atrioventricular reentrant tachycardia (AVRT) with an accessory pathway. Children with wide QRS complex tachycardia may p

Introduction: Postural tachycardia syndrome (POTS) is a disorder of orthostatic intolerance characterized by excessive tachycardia of unknown etiology. Our objective in this study was to evaluate the correlation between C-fiber involvement as shown by skin biopsy and adrenergic cardiac metaiodobenzylguanadine (MIBG) uptake in POTS patients. Methods: Skin biopsies of 84 patients with POTS were examined by Protein Gene Product 9.5 (PGP9.5) immunohistochemistry and were compared with MIBG myocardial scintigraphy imaging data. Results: Mean intraepidermal nerve fiber (IENF) density was in the lower normal age-adjusted range, 7.2 ± 2.9/mm (normal ≥7/mm), and was slightly below the normal range in 45% of POTS patients. MIBG uptake was reduced in 21% of patients. Low IENF density correlated with reduced cardiac MIBG uptake (r = 0.39, P = 0.001). Conclusions: A subset of neuropathic POTS patients may harbor mild small fiber neuropathy with abnormalities of unmyelinated nerve fibers in the skin ...

Atrial tachycardia is a type of heart rhythm problem in which the hearts electrical impulse comes from an ectopic pacemaker (that is, an abnormally located cardiac pacemaker) in the upper chambers (atria) of the heart, rather than from the sinoatrial node, the normal origin of the hearts electrical activity. Atrial tachycardias can exhibit very regular (consistent) heart rates ranging typically from 140 to 220 beats per minute.[1] As with any other form of tachycardia (rapid heart beat), the underlying mechanism can be either the rapid discharge of an abnormal focus, the presence of a ring of cardiac tissue that gives rise to a circle movement (reentry),[2] or a triggered rapid rhythm due to other pathological circumstances (as would be the case with some drug toxicities, such as digoxin toxicity). Atrial tachycardia is a risk factor for atrial fibrillation, as the rapid rhythm can trigger or degrade into the lack of a rhythm.[3] All atrial tachycardias are by definition supraventricular ...

Providing information and support about postural orthostatic tachycardia syndrome (PoTS) for sufferers, medical professionals, family, and friends - aiming to raise awareness.

Providing information and support about postural orthostatic tachycardia syndrome (PoTS) for sufferers, medical professionals, family, and friends - aiming to raise awareness.

The morphology of the tachycardia depends on its cause and the origin of the re-entry electrical circuit in the heart. In monomorphic ventricular tachycardia, the shape of each heart beat on the ECG looks the same because the impulse is either being generated from increased automaticity of a single point in either the left or the right ventricle, or due to a reentry circuit within the ventricle. The most common cause of monomorphic ventricular tachycardia is scarring of the heart muscle from a previous myocardial infarction (heart attack). This scar cannot conduct electrical activity, so there is a potential circuit around the scar that results in the tachycardia. This is similar to the re-entrant circuits that are the cause of atrial flutter and the re-entrant forms of supraventricular tachycardia. Other rarer congenital causes of monomorphic VT include right ventricular dysplasia, and right and left ventricular outflow tract VT. Polymorphic ventricular tachycardia, on the other hand, is most ...

Objectives: Patients with postural tachycardia syndrome (POTS) often appear anxious and report inattention. Patients with POTS were formally assessed for psychiatric disorders and inattention and compared with patients with attention deficit hyperactivity disorder (ADHD) and control subjects.. Methods: Patients with POTS (n = 21), ADHD (n = 18) and normal control subjects (n = 20) were assessed for DSM-IV psychiatric disorders and completed a battery of questionnaires that assessed depression, anxiety and ADHD characteristics.. Results: Patients with POTS did not have an increased prevalence of major depression or anxiety disorders, including panic disorder, compared with the general population. Patients with POTS had mild depression. They scored as moderately anxious on the Beck Anxiety Inventory but did not exhibit a high level of anxiety sensitivity. Patients with POTS scored significantly higher on inattention and ADHD subscales than control subjects. These symptoms were not present during ...

What is ventricular tachycardia? Ventricular tachycardia is an arrhythmia represented by rapid heartbeats that originate in the ventricles. During ventricular tachycardia the heart may beat inefficiently and as a result the blood pressure may decrease very much. This type of tachycardia may lead to life-threatening arrhythmias and cardiac arrest. Treatment options for ventricular tachycardias include medications, catheter ablation or placement of an implantable cardioverter-defibrillator (ICD).

Background: When noninvasive studies remain negative, the diagnosis of unexplained tachycardia in the youth is a dilemma. Invasive electrophysiological study (EPS) requires generally anesthesia and hospitalization in young children. The purpose of the study was to determine the factors of negativity of transesophageal EPS in children and teenagers complaining of tachycardia.. Population: 267 children, aged from 6 to 19 years, mean 15±3, complained of tachycardia and had a normal ECG in sinus rhythm.. Methods: Transesophageal EPS was performed in out-patient clinic without sedation but in presence of parents; EPS consisted of atrial pacing and atrial stimulation with 1 and 2 extrastimuli in control state and after isoproterenol. Long-term Holter monitoring was systematic in patients with negative EPS.. Results: Supraventricular tachycardia (SVT)(AV node re-entrant tachycardia, AV re-entrant tachycardia, atrial tachycardia) was induced in 146 patients (group I) and EPS remained negative in 121 ...

Ventricular tachycardia is the term for a fast heart rate that begins in the ventricles, the lower section of the heart. It leads to a fast yet regular heart rhythm. However, it can lead to ventricular fibrillation, which is a fast yet irregular heart beat. With this, the heart may beat so quickly and irregularly that it actually stops pumping blood. This can lead to sudden cardiac arrest and death. However, with treatment ventricular tachycardia can be handled and wont develop into ventricular fibrillation.. At times, especially when it develops in children and teens, it can be difficult if not impossible to identify the cause of ventricular tachycardia. However, in most cases, some sort of heart disease is the base cause. This may include a congenital heart defect, a previous heart attack, myocarditis, or dilated or hypertrophic cardiomyopathy. At times, ventricular tachycardia develops after a person has heart surgery. Some medications can also cause ventricular tachycardia. These include ...

Introduction. Neonatal tachycardia is defined as a resting heart rate (HR) of 182 beats per minute (bpm) when the baby is not crying.1,2 Supraventricular tachycardia (SVT) is the most common tachyarrhythmia in the neonatal period and usually presents with a HR of more than 200bpm and with a narrow QRS complex. In 80% of patients, it originates through a mechanism of retrograde conduction via an accessory pathway between the ventricle and the atrium, with an abrupt onset and termination. In 15% of patients it results from atrial ectopic beats, while 5% of cases correspond to re-entrant nodal tachycardias.3,4 Approximately half of these patients present with heart failure, with suspicion of sepsis in some cases. The tachycardia may be detected in the foetal period, most frequently between weeks 28 and 33 of gestation, possibly manifesting with hydrops.5. The key element in the management of haemodynamically unstable patients with tachycardia is electric cardioversion. However, in stable patients, ...

TY - JOUR. T1 - Use of the impella™ microaxial blood pump for ablation of hemodynamically unstable ventricular tachycardia. AU - Abuissa, Hussam. AU - Roshan, John. AU - Lim, Bernard. AU - Asirvatham, Ssamuel J.. PY - 2010/4. Y1 - 2010/4. N2 - Impella™ for VT Ablation. Ablation for ventricular tachycardia remains a challenge with suboptimal procedural success rates. One of the major causes of difficulty is precipitous hypotension when ventricular tachycardia is induced precluding even rapid mapping of the arrhythmia. We report the successful use of the Impella™ microcirculatory axial blood flow pump in 3 patients with hemodynamically unstable ventricular tachycardia that allowed successful completion of the procedure. In these 3 patients, there was no evidence of Impella™-related valvular disturbance, iatrogenic ventricular arrhythmias, or interference with mapping and ablation catheter movement. (J Cardiovasc Electrophysiol, Vol. 21, pp. 458-4, April 2010). AB - Impella™ for VT ...

TY - JOUR. T1 - Optimal surface electrocardiogram lead for identification of the mechanism of supraventricular tachycardia in children. AU - Liberman, Leonardo. AU - Pass, Robert H.. AU - Starc, Thomas J.. PY - 2008/1/1. Y1 - 2008/1/1. N2 - OBJECTIVE: Although supraventricular tachycardia (SVT) can be identified from any lead of the bedside monitor, the mechanism of tachycardia is not always obvious. We analyzed the 12-lead electrocardiogram (ECG) in SVT of pediatric patients with different mechanisms of SVT to determine if there is a consistent optimal lead for rhythm identification. METHODS: Twelve-lead ECGs during SVT were available for retrospective analysis in 54 patients. The tachycardia mechanism was determined either by intracardiac or transesophageal recording, or after cardioversion analysis of atrial flutter or fibrillation. Blinded analysis of each separate lead of the 12-lead ECG was done to determine the best lead to diagnose the mechanism of tachycardia. For statistical analysis, ...

A closed-heart method for treating ventricular tachycardia in a myocardial infarct patient afflicted with ventricular tachycardia is disclosed. The method comprises, first, defining a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) of the patient, and then ablating the thin layer of spared myocardial tissue by a closed-heart procedure with an ablation catheter. Apparatus for carrying out the method is also disclosed, Also disclosed is a method for prognosing the likelihood of ventricular tachycardia occuring in a myocardial infarct patient not previously diagnosed as afflicted with ventricular tachycardia. The method comprises detecting a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) in the patient.

A closed-heart method for treating ventricular tachycardia in a myocardial infarct patient afflicted with ventricular tachycardia is disclosed. The method comprises, first, defining a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) of the patient, and then ablating the thin layer of spared myocardial tissue by a closed-heart procedure with an ablation catheter. Apparatus for carrying out the method is also disclosed. Also disclosed is a method for prognosing the likelihood of ventricular tachycardia occuring in a myocardial infarct patient not previously diagnosed as afflicted with ventricular tachycardia. The method comprises detecting a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) in the patient.

STARS PO Box 175 Stratford-upon-Avon Warwickshire CV37 8YD +44 (0) 1789 450 564 [email protected] www.stars.org.uk Postural Tachycardia Syndrome (PoTS) This information sheet is designed for patients who have been diagnosed with postural tachycardia syndrome (PoTS). It contains information on the diagnosis, treatment and management of PoTS. Postural tachycardia syndrome (PoTS) is an abnormality of functioning of the autonomic (involuntary) nervous system. It is defined as an increase in heart rate of over 30 beats per minute (or to higher than 120 beats per minute) (40 bpm in those age 12-19) when standing upright. Typically there is no postural fall in blood pressure, although fainting (syncope) can occur (see below). The symptoms can vary (see below) but mostly involve orthostatic intolerance (the inability to withstand the upright posture) and are often made worse by various daily activities, e.g., modest physical exertion or food. It is a disorder which is slowly being recognised by the ...

Re-entrant atrial tachycardia is usually seen after cardiac surgery or catheter ablation with linear lesions that result in islets of scars. In patients with structurally normal hearts, atrial tachycardia is associated with a low mortality rate. Objectives: Ann Intern Med. My cardiologist now tells me this is common in about 50% of patients and is most likely a temporary issue. It is usually a self-limiting condition, but can cause serious haemodynamic deterioration in the immediate postoperative phase .JET is a narrow QRS complex tachycardia, usually with atrioventricular dissociation and slower atrial than ventricular rate. JAMA. I recently (August 2018) had my 1st ablation at TCA in Austin TX. 2017;92(1):98-105. Arrhythmias are common after cardiac surgery such as coronary artery bypass grafting surgery. Dual-loop circuits in postoperative atrial macro re-entrant tachycardias. 2007;89:91-5. reported that the most common site of focal ATs after AF ablation was the previously isolated PV. ...

Ventricular tachycardia is a very rare fetal arrhythmia accounting for fewer than 2% of fetal tachycardias. We describe a fetus presenting at 30 weeks gestation with ventricular tachycardia at a rate of 220 beats per min and fetal hydrops. The tachycardia was unresponsive to flecainide but was co...

Looking for Sinus tachycardia? Find out information about Sinus tachycardia. see arrhythmia arrhythmia , disturbance in the rate or rhythm of the heartbeat. Various arrhythmias can be symptoms of serious heart disorders; however,... Explanation of Sinus tachycardia

Looking for online definition of auricular tachycardia in the Medical Dictionary? auricular tachycardia explanation free. What is auricular tachycardia? Meaning of auricular tachycardia medical term. What does auricular tachycardia mean?

The heart has its own electrical system that controls the heart rate and contraction of the heart, which pumps blood throughout the body. Normal heart rates vary based on a persons age.. A rapid heart rate can be caused by many different factors. Sometimes tachycardia is the bodys response to a trigger like fever, anxiety, exercise, or pain, just to name a few. It also can be due to an abnormal rhythm in the heart (called a dysrhythmia), such as supraventricular tachycardia (SVT).. Congenital heart abnormalities, high blood pressure, medications, dehydration, caffeine, smoking, and recreational drug use also can cause tachycardia. Tachycardias can lead to dizziness, shortness of breath, chest pain, fainting, and heart palpitations (a sensation of feeling the heart beat fast or irregularly). Some tachycardias are life threatening and can lead to blood clots, causing a stroke, heart failure, and sudden death.. ...

The subjects are patients with COI and adult healthy volunteers of similar age, gender, and body mass.. Participation in this protocol is offered to people 18 years old or older, independently of gender, race, age, ethnicity, religion, or any other demographic or sociopolitical classifications.. The subjects in the second off-site study are family members of the identified proband. Participation in this protocol is offered to people 18 years old or older, independently of gender, race, ethnicity, religion, or any other demographic or sociopolitical classifications.. In the second off-site study, participation is also offered to subjects 10 to 17 years old, provided that appropriate informed consent/assent has been given by the subject and his/her parent or guardian.. EXCLUSION CRITERIA:. Age: Minors younger than 18 years old are excluded, except for the off-site study. Advanced age does not constitute an exclusion criterion. For subjects more than 55 years old, carotid Doppler studies are done, ...

This is an irregular narrow complex rhythm. The first 2 sinus P waves (S) are of the same morphology with a PRI of about 0.20 sec and a rate of about 60 bpm (lead II). The tachycardia starts with a premature complex with different P wave morphology (blue arrow). The tachycardia is best observed in V1. The rate tends to increase (107 to about 170 bpm) then there is a non-conducted P wave (first red arrow). The first conducted P has a shorter PRI compared to the previous PRI. The tachycardia resumes with 1:1 conduction then a non-conducted P wave is seen (second red arrow). Thus, this is atrial tachycardia with a block (type I block). ...

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Researchers discover autoantibodies in POTS patients that may account for increased circulation of norepinephrine and tachycardia upon standing.

TY - JOUR. T1 - Mode of induction of ventricular tachycardia and prognosis in patients with coronary disease. T2 - The multicenter unsustained tachycardia trial (MUSTT). AU - Piccini, Jonathan P.. AU - Hafley, Gail E.. AU - Lee, Kerry L.. AU - Fisher, John D.. AU - Josephson, Mark E.. AU - Prystowsky, Eric N.. AU - Buxton, Alfred E.. PY - 2009/8/1. Y1 - 2009/8/1. N2 - Mode of Induction in MUSTT. Introduction: Programmed stimulation is an important prognostic tool in the evaluation of patients with an ejection fraction ≤40% after myocardial infarction. Many believe that ventricular tachycardia (VT) requiring 3 ventricular extrastimuli (VES) for induction is less likely to occur spontaneously and has less predictive value. However, it is unknown whether the mode of VT induction is associated with long-term prognosis. Methods and Results: We analyzed a cohort of 371 patients enrolled in MUSTT who had inducible monomorphic VT and who were not treated with antiarrhythmic drugs or an implantable ...

POTS is a relaytively common cause of disabilty in young people, characterized by symptoms associated with fast heartbeat (tachycardia) upon standing.

With monomorphic ventricular tachycardia all of the QRS waves will likely be symmetrical. Every ventricular impulse is being created from the identical place in the ventricles thus all of the QRS

Case report A 31 year old female was admitted to A/E 6 hours after ingestion of 90 bupropion tablets (13.5g), with alcohol. Initially, the patient was conscious but disorientated, GCS 12/15, agitated, tachycardia (124 beats per minute), raised blood pressure (180/96), tremor, dystonia, increased muscle tone with brisk tendon reflexes and clonus. Activated charcoal was administered but the patient had a convulsion and aspirated. She deteriorated further with recurrent seizures, GCS 5/15, BP 120/60 and heart rate 120 beats per minute and required intubation, ventilation and sedation with diazepam. The ECG showed broad complex tachycardia, which reverted to normal sinus rhythm after treatment with adenosine 12mg iv (Figure 1). The patient was ventilated for 2 days during which she remained hypotensive and had extrapyramidal signs. She was extubated after 3 days. Discussion Bupropion, an atypical antidepressant is structurally similar to amphetamine and diethylpropion.2 It was originally approved ...

Dive into the research topics of Outer loop and isthmus in ventricular tachycardia circuits: Characteristics and implications. Together they form a unique fingerprint. ...

During wide complex tachycardia (heart rate , 100/min, QRS , 0.12 sec) the differentiation between supraventricular and ventricular origin of the arrhythmia is important to guide therapy. Several algorithms have been developed to aid in this differentiation (below). It is important to keep in mind that a good estimate of VT versus SVT can be made based on the clinical vignette: ...

Tachycardia induced cardiomyopathy is a potentially lethal cause of heart failure generally because of atrial tachycardia and less frequently ventricular tachycardia. We present two cases of Marines with severe traumatic blast injuries secondary to i

There are 2 main new findings in the present study. The first one is that, after removing autonomic influences, inhibition of endogenous NO synthesis with L-NMMA results in significant bradycardia. This effect is not explained by its pressor effect or baroreflex responses, as the same increase in SBP with phenylephrine did not reduce HR during autonomic blockade. These data show that NO has a tonic effect on human HR in vivo, and that this effect is independent of the autonomic control of HR. It is well known that NO is produced within the heart not only by endothelial cells, but also by myocytes.20,21 It has been proposed that NO has a role in the regulation of cardiac function both under normal and pathological conditions.11,22,23 NOS inhibition was previously shown to reduce HR in patients with transplanted, and presumably denervated, hearts.24 Our findings indicate for the first time that endogenous NO tonically regulates HR in healthy subjects. This is also in agreement with studies in ...

Symptomatic falls in blood pressure after standing or eating are a frequent clinical problem, particularly in the elderly. The symptoms are often due to cerebral hypoperfusion and include generalized weakness, dizziness or lightheadedness, visual blu

The heartbeat is controlled by the electrical system of the heart. This system is made up of several parts that tell the muscle of the heart when to contract. The SA node starts the heartbeat, causing the atria, or upper chambers of the heart, to contract. The signal then travels through the AV node, bundle of His, bundle branches, and Purkinje fibers. This causes the ventricles, the lower chambers of the heart, to contract. This organized flow of electrical signals produces a normal heartbeat. Normal heartbeats can be seen in an Electrocardiogram or ECG.. Focal Ventricular Tachycardia is a heart arrhythmia, or irregular heartbeat. During focal ventricular tachycardia, accelerated abnormal electrical pulses in the lower chambers, or ventricles, disrupt the normal firing of the SA node, causing the heart to beat rapidly. The abnormal signals originate in a specific area in the ventricles called a focus. This irregular heartbeat can be seen on an electrocardiogram.. A rapid heartbeat does not give ...

Ventricular tachycardia (VT) is a type of regular, fast heart rate that arises from improper electrical activity in the ventricles of the heart.. ​. Ventricular tachycardia may last for only a few seconds, or it can last for much longer. You may feel dizzy or short of breath, or have chest pain.. Although a few seconds may not result in problems, longer periods are dangerous.. ​. Sometimes, ventricular tachycardia can cause your heart to stop (cardiac arrest), which is a life-threatening medical emergency.. ​. Many different conditions can cause ventricular tachycardia. Its important to get a prompt, accurate diagnosis and appropriate care.. ​. The best way to prevent ventricular tachycardia is to treat or eliminate risk factors that may lead to heart disease. If you already have heart disease, follow your treatment plan and a heart-healthy lifestyle.. See your doctor if you have any problems with the heartbeat. In some cases, urgent care is needed. ...

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Ventricular Tachycardia Treatment in Frisco - Ventricular Tachycardia is the issue related to the heart. If we talk about Texas, many hospitals provide the be