Tachycardia, also called tachyarrhythmia, is a heart rate that exceeds the normal resting rate. In general, a resting heart rate over 100 beats per minute is accepted as tachycardia in adults. Heart rates above the resting rate may be normal (such as with exercise) or abnormal (such as with electrical problems within the heart). The upper threshold of a normal human resting heart rate is based on age. Cutoff values for tachycardia in different age groups are fairly well standardized; typical cutoffs are listed below: 1-2 days: Tachycardia > 159 beats per minute (bpm) 3-6 days: Tachycardia >166 bpm 1-3 weeks: Tachycardia >182 bpm 1-2 months: Tachycardia >179 bpm 3-5 months: Tachycardia >186 bpm 6-11 months: Tachycardia >169 bpm 1-2 years: Tachycardia >151 bpm 3-4 years: Tachycardia >137 bpm 5-7 years: Tachycardia >133 bpm 8-11 years: Tachycardia >130 bpm 12-15 years: Tachycardia >119 bpm >15 years - adult: Tachycardia >100 bpm Heart rate is considered in the context of the prevailing clinical ...
Mechanistic studies of human heart failure are complicated by the prolonged time course of development of the disease, the technical challenges of isolating cardiac tissue or cells from explanted hearts, the inability to investigate the early time course of cellular alterations, and the lack of control over experimental conditions. Thus, it is fortunate that the canine tachycardia-induced heart failure model so closely reproduces the known hemodynamic and ionic changes that have been identified in human hearts. The present findings indicate that, in addition to the electrophysiological changes noted in earlier studies, significant alterations in Ca2+ handling occur in isolated myocytes from failing hearts, following the general pattern of human studies. Through biochemical and functional measurements in the same hearts, we have found strong evidence in support of the hypothesis that the induction of heart failure triggers a shift in the balance of cytosolic Ca2+ extrusion mechanisms from SR Ca2+ ...
Successful Ablation for Atrial Tachycardia Originated from Sinus Venosa with Tachycardia-Induced Cardiomyopathy. . Biblioteca virtual para leer y descargar libros, documentos, trabajos y tesis universitarias en PDF. Material universiario, documentación y tareas realizadas por universitarios en nuestra biblioteca. Para descargar gratis y para leer online.
TY - JOUR. T1 - Tachycardia in Post-Infarction Hearts. T2 - Insights from 3D Image-Based Ventricular Models. AU - Arevalo, Hermenegild. AU - Plank, Gernot. AU - Helm, Patrick. AU - Halperin, Henry R. AU - Trayanova, Natalia. PY - 2013/7/2. Y1 - 2013/7/2. N2 - Ventricular tachycardia, a life-threatening regular and repetitive fast heart rhythm, frequently occurs in the setting of myocardial infarction. Recently, the peri-infarct zones surrounding the necrotic scar (termed gray zones) have been shown to correlate with ventricular tachycardia inducibility. However, it remains unknown how the latter is determined by gray zone distribution and size. The goal of this study is to examine how tachycardia circuits are maintained in the infarcted heart and to explore the relationship between the tachycardia organizing centers and the infarct gray zone size and degree of heterogeneity. To achieve the goals of the study, we employ a sophisticated high-resolution electrophysiological model of the infarcted ...
A pacing system provided with a mode switching feature and ventricular rate regularization (VRR) function adapted to stabilize or regularize ventricular heart rate during chronic or paroxysmal atrial tachyarrhythmia. In a preferred embodiment, the pacing system nominally operates in an atrial synchronized pacing mode such as DDD or DDDR pacing mode. In response to detection of atrial rhythm characteristics consistent with an atrial tachyarrhythmia, e.g., atrial fibrillation, a mode switch into a non-atrial synchronized, ventricular rate regularization pacing mode, e.g. DDIR or VDIR pacing mode, is made. If the VRR function is programmed on, the ventricular pacing rate based upon a rate responsive sensor derived ventricular pacing rate modulated on a beat by beat basis by preceding intrinsic or paced ventricular events, the stability of the intrinsic ventricular heart rate, and any atrial pace events to regularize the ventricular pacing rate. The pacing system may also be permanently programmed to the
The freeMD virtual doctor has found 32 conditions that can cause Abdominal Tenderness and Tachycardia. There are 6 common conditions that can cause Abdominal Tenderness and Tachycardia. There are 4 somewhat common conditions that can cause Abdominal Tenderness and Tachycardia. There are 7 uncommon conditions that can cause Abdominal Tenderness and Tachycardia. There are 15 rare conditions that can cause Abdominal Tenderness and Tachycardia.
This page includes the following topics and synonyms: Tachycardia due to Medications, Drug Induced Tachycardia, Toxin Induced Tachycardia.
The study of conversion of AF to sinus rhythm provides information on changes associated with the resumption of atrial contraction and AV synchrony leading to a regularized, rate-controlled ventricular response. Previous studies have documented improvement in cardiac output ([75]) and cardiopulmonary exercise testing variables of ventricular rate control, maximal oxygen uptake and anaerobic threshold ([76, 77]) after cardioversion to sinus rhythm. More recently, a small, prospective study of patients with a diagnosis of "idiopathic" dilated cardiomyopathy and chronic AF reported a significant increase in left ventricular ejection fraction after pharmacologic or electrical cardioversion to sinus rhythm ([78]). Whether improvement in cardiac function was due to restoration of atrial systolic function and AV synchrony versus reversal of an underlying cardiomyopathy associated with rapid and irregular ventricular rates during AF was not resolved by this study. Subsequent study of atrial and ...
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The rhythm is a regular WCT ( = Wide-Complex Tachycardia) at ~ 210/minute without sign of atrial activity. Dr. Smith has reviewed the principal differential diagnosis. I dont think QRS duration is particularly helpful here, given that the patient is a 12-year old child (for whom normal QRS duration is a bit less than in adults). So the QRS is definitely wide, though not excessively so - but probably overlaps most entities to be considered. What strikes me - is the obvious suggestion of bifascicular block (RBBB/LAHB), though of atypical morphology. In an otherwise healthy child (who does not have congenital heart disease or other structural heart abnormality) - one would expect a more typical RBBB morphology with a clear triphasic appearance with taller right-rabbit ear in lead V1, instead of the atypical picture we see here in V1,V2,V3. Considering this and integrating it with the important Learning Points Dr. Smith emphasizes (ie, that fascicular VT often occurs in otherwise normal hearts) - ...
Most of us (assuming no pediatric cardiologists are listening) do not see a large number of pediatric arrhythmias (certainly far less than weve all seen in adults). As a result - level of comfort in both diagnosis and management tends to be less than that attained with adult arrhythmias. As wonderfully emphasized by Dr. Smith by the 2 pediatric arrhythmia examples shown here - accurate determination of rate is CRITICAL in the assessment of pediatric arrhythmias. For this determination - I have found use of the "Every-other-Beat" (or sometimes, "Every-third-Beat") method a user-friendly way to rapidly and accurate determine heart rate for any regular fast rhythm. While most of us recall, "300- 150 - 100 - 75 - 60 - 50" as the answers for rate calculation when the R-R interval of a regular tachycardia is 1, 2, 3, 4, 5 or 6 large boxes, respectively - this becomes problematic with very fast rates. Thats because small differences in measurements may translate into significant under- or ...
Improved methods and devices perform tachycardia detection and anti-tachycardia pacing (ATP) to convert a tachycardia (e.g., VT or AT) to normal sinus rhythm. According to one embodiment, an anti-tachycardia pacing method includes sensing, during sinus rhythm, first and second cardiac signals at first and second sites, respectively, in a patients heart. The first and second sites include left and right ventricles or left and right atria, for example. The method further includes sensing third and fourth cardiac signals at the first and second sites, respectively, during a tachycardia (e.g., ventricular tachycardia or atrial tachycardia). The cardiac signals are processed to provide respective values. One or more anti-tachycardia pacing pulses are delivered at the site closest to the reentrant circuit based on a comparison of a first ratio of the first and third values and a second ratio of the second and fourth values. Unipolar sensing of the cardiac signals may be employed by, for example,
Tachycardia: Abnormally rapid heartbeat, usually with a HEART RATE above 100 beats per minute for adults. Tachycardia accompanied by disturbance in the cardiac depolarization (cardiac arrhythmia) is called tachyarrhythmia.
What is tachycardia arrhythmia? Learn about tachycardia arrhythmia (fast heartbeat), including causes, risk factors, symptoms, diagnosis and treatment from the cardiology experts at Mercy Health.
Fascinating case with a number of interesting insights to learn. Diagnosis of alternate preexcitation on the last tracing is subtle, but important to recognize. The 2 other thoughts I considered in my differential diagnosis for this last ECG were: i) electrical alternans; and ii) ventricular bigeminy with fusion from end-diastolic PVCs - but neither "fit" (thats because we see DELTA waves in several leads on this tracing that provide the diagnosis). Otherwise - the T wave peaking we see on the tachycardia ECGs is extremely interesting. I have often seen T wave peaking with tachycardia (and on stress tests) in otherwise healthy young adults - but I dont expect ST elevation. In addition, this patient was apparently markedly dehydrated on each occasion when the ECG was done. Hyperkalemia (perhaps secondary to volume contraction) not uncommonly leads to T wave peaking with recurrent episodes, but again supposedly not associated with ST elevation (Wonder what serum K+ values were in this patient ...
Tachycardia refers to a fast resting heart rate, usually over 100 beats per minute. Tachycardia can be dangerous, depending on its underlying cause and on
BACKGROUND: Catheter ablation (CA) therapy is the first choice treatment in adults with heart rhythm disturbances. The arrhythmias in adults are mainly conditioned by coronary disease. Etiology of arrhythmias in children is mostly associated with inherited heart disorders. According to the current guidelines the CA is widely used in children, indicating the need to make it more achievable in pediatric population. AIM: To assess efficacy and safety of CA in children with different types of arrhythmias on the initial learning curve at newly built Ablation Center in The Independent Pediatric Hospital of Medical University of Warsaw ...
Most arrhythmia including Tachycardia are not serious However, any abnormality associated with the heart can quickly develop into a serious, or even life
An interesting technicality in the use of the PERC rule to rule out pulmonary embolism is the tachycardia component - it asks not whether the patient is tachycardic at the time of the application of the rule, or whether tachycardia was sustained throughout the emergency department stay, but instead whether the patient had (as described by Jeff Kline in his great review article on PE diagnosis and risk stratification): "3. Pulse ,100 beats/min during entire stay in ED". Meaning, even transient tachycardia may suggest a life-threatening diagnosis, even if it resolves while the patient is in the emergency department, and were probably PERCing out a whole bunch of patients inappropriately, at least according to Kline (who, notably, testifies a whole bunch as an expert witness in cases of missed pulmonary emboli).. I recently had a handful of patients in whom concerning blood pressures were measured and documented, which then resolved when vital signs were re-checked or after a small quantity of ...
You are working one evening in the emergency department when a 60-something year old female is slotted for a room. Her chief complaint? "Fever, weakness, vomiting". Seeing that her triage heart rate was 157, you leave your granola bar where it is and immediately walk into the room to assess her. You see an elderly-appearing female in moderate respiratory distress. Her temperature is 38.2, blood pressure is 125/87, RR is 32, oxygen saturation is 93% on 5L NC. She has a history of a bone-marrow transplant and is chronically immunosuppressed. She endorses poor PO intake and several episodes of emesis over the last few days. She says that she came in today when she developed some shortness of breath as well. She denies any chest pain or palpitations. On exam, her mucous membranes are dry, her abdomen non-tender, and her breath sounds are decreased in the right base. You are a bit disturbed by the looks of her rhythm strip on the monitor so you get a 12-lead EKG ...
Learn more about Tachycardia at Reston Hospital Center DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...
Learn more about Tachycardia at Grand Strand Medical Center DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...
Tachycardia is a condition where a persons heart is beating abnormally fast. When we become more active our heart rate increases to meet the increased dem
Atrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics (costs for program #250319) ✔ University Hospital Halle (Saale) ✔ Department of Cardiology and Angiology (Department of Internal Medicine III) ✔ BookingHealth.com
Atrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics (costs for program #29263) ✔ Academic Hospital Hildesheim ✔ Department of Cardiology, Angiology, Rhythmology and Intensive Care Medicine ✔ BookingHealth.com
Tachycardia is a rapid heart rate caused by a problem in the hearts electrical system. Find out more about this common heart rhythm disorder (arrhythmia).
A pacemaker-mediated tachycardia (PMT) can be defined as any condition in which a pacemaker paces the ventricles at rates that are inappropriately fast. This can be due to (1) a rate response setting that is too sensitive, (2) tracking of atrial noise (such as what may occur with electromagnetic interference), (3) inappropriate pacemaker mani...
A heart rate of more than 100 beats per minute (BPM) in adults is called tachycardia. Whats too fast for you may depend on your age and physical condition.
Rapid and abnormal increase in heart rate more than 100 beats a minute in rest is tachycardia. It is caused by abnormal electrical impulses originated in the heart chambers with a physiological or pathological background.
Its been a while since I been on here, but I must say life is good, I am truly blessed. My pvcs are almost completely gone, I may feel only a couple a week and I havent had a tachycardia attack in over three months, I must say again God is good. I was once afraid to live on my own because if my heart issues, but I recently moved into my own house three weeks ago. I posted a while back about the doctor putting me on Mirtazapine for anxiety and weight gain, it has really helped me alot, I started off at 111lbs, I am now 134lbs ...
Question - Suffering from SVT tachycardia. Prescribed with metroprolol. Is it safe to go for skiing?. Ask a Doctor about diagnosis, treatment and medication for Increased heart rate, Ask a Cardiologist
how high can an axiety attact drive ones HR up? to 110, 120, 130?? I fhigh, one will confuse the adrenaline caused high HR that stops in a few minutes with tachycardia. More and more I believe, aft...
A disruption in the normal electrical impulses controlling the pumping action of the heart causes tachycardia, according to Mayo Clinic. Many different factors cause these disruptions and the related...
Had cathetor ablation following a 189 heart beat which I could not control (prior to that over a number of years had, say 120 beats - I could stop by holding my breath a few times and it would disappe
I have been experiencing a fast, pounding heart rate for over a month now. It gets worse when I stand up. I get light headed, very short of breath, dizzy, weak, and feel as if Im going to faint. I wen...
I have been wearing a chest hr monitor for the last week and have been concerned that I seem to be tachycardiac in the afternoons. My resting heart...
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Abnormal heartbeat is a more common problem than people care to understand. Scientifically known as an Arrhythmia or tachycardia, based on the type, abnormal...
Are Zinadol Side Effects Putting Your Health at Risk? | Jan 10, 2018 Check these Zinadol side effect reports: A 41-year-old female patient was diagnosed with NA, treated with ZINADOL and reported blood pressure increased,tachycardia. Dosage: Unk. Patient was hospitalized.
23 y/o male presents to the ER with tachycardia and hypotension after a high speed MVA. http://forum.usmleforumland.com/filedata/fetch?id=305
Objectives. The purpose of this study was to determine the predictors of electrically induced ventricular tachycardia in a large sample of patients with unexplained syncope and to examine the value of the signal-averaged electrocardiogram (ECG) in those patient subsets with varying pretest probability of ventricular tachycardia.. Background. In patients with unexplained syncope, electrophysiologic study can provide important diagnostic information, such as inducibility of ventricular tachycardia. The signal-averaged ECG can predict inducible ventricular tachycardia, but its utility has not been prospectively studied in a large group of patients with unexplained syncope.. Methods. At six hospitals, 189 consecutive patients with unexplained syncope underwent signal-averaged ECG and electrophysiologic studies.. Results. Ventricular tachycardia was induced in 28 patients (15%). Univariate predictors of ventricular tachycardia included history of previous myocardial infarction, reduced left ...
The Postural Tachycardia Syndrome (POTS) is the most common disorder seen in autonomic clinics. Cardinal hemodynamic feature of this chronic and debilitating disorder of orthostatic tolerance is an exaggerated orthostatic tachycardia (≥30 bpm increase in HR with standing) in the absence of orthostatic hypotension. There are multiple pathophysiological mechanisms that underlie POTS. Some patients with POTS have evidence of elevated sympathoneural tone. This hyperadrenergic state is likely a driver of the excessive orthostatic tachycardia. Another common pathophysiological mechanism in POTS is a hypovolemic state. Many POTS patients with a hypovolemic state have been found to have a perturbed renin-angiotensin-aldosterone profile. These include inappropriately low plasma renin activity and aldosterone levels with resultant inadequate renal sodium retention. Some POTS patients have also been found to have elevated plasma angiotensin II (Ang-II) levels, with some studies suggesting problems with
Wide QRS tachycardia may be due to ventricular tachycardia (VT), supraventricular tachycardia (SVT) with aberrant conduction, or atrioventricular reentrant tachycardia (AVRT) with an accessory pathway. Children with wide QRS complex tachycardia may p
Introduction: Postural tachycardia syndrome (POTS) is a disorder of orthostatic intolerance characterized by excessive tachycardia of unknown etiology. Our objective in this study was to evaluate the correlation between C-fiber involvement as shown by skin biopsy and adrenergic cardiac metaiodobenzylguanadine (MIBG) uptake in POTS patients. Methods: Skin biopsies of 84 patients with POTS were examined by Protein Gene Product 9.5 (PGP9.5) immunohistochemistry and were compared with MIBG myocardial scintigraphy imaging data. Results: Mean intraepidermal nerve fiber (IENF) density was in the lower normal age-adjusted range, 7.2 ± 2.9/mm (normal ≥7/mm), and was slightly below the normal range in 45% of POTS patients. MIBG uptake was reduced in 21% of patients. Low IENF density correlated with reduced cardiac MIBG uptake (r = 0.39, P = 0.001). Conclusions: A subset of neuropathic POTS patients may harbor mild small fiber neuropathy with abnormalities of unmyelinated nerve fibers in the skin ...
Atrial tachycardia is a type of heart rhythm problem in which the hearts electrical impulse comes from an ectopic pacemaker (that is, an abnormally located cardiac pacemaker) in the upper chambers (atria) of the heart, rather than from the sinoatrial node, the normal origin of the hearts electrical activity. Atrial tachycardias can exhibit very regular (consistent) heart rates ranging typically from 140 to 220 beats per minute.[1] As with any other form of tachycardia (rapid heart beat), the underlying mechanism can be either the rapid discharge of an abnormal focus, the presence of a ring of cardiac tissue that gives rise to a circle movement (reentry),[2] or a triggered rapid rhythm due to other pathological circumstances (as would be the case with some drug toxicities, such as digoxin toxicity). Atrial tachycardia is a risk factor for atrial fibrillation, as the rapid rhythm can trigger or degrade into the lack of a rhythm.[3] All atrial tachycardias are by definition supraventricular ...
Providing information and support about postural orthostatic tachycardia syndrome (PoTS) for sufferers, medical professionals, family, and friends - aiming to raise awareness.
Providing information and support about postural orthostatic tachycardia syndrome (PoTS) for sufferers, medical professionals, family, and friends - aiming to raise awareness.
The morphology of the tachycardia depends on its cause and the origin of the re-entry electrical circuit in the heart. In monomorphic ventricular tachycardia, the shape of each heart beat on the ECG looks the same because the impulse is either being generated from increased automaticity of a single point in either the left or the right ventricle, or due to a reentry circuit within the ventricle. The most common cause of monomorphic ventricular tachycardia is scarring of the heart muscle from a previous myocardial infarction (heart attack). This scar cannot conduct electrical activity, so there is a potential circuit around the scar that results in the tachycardia. This is similar to the re-entrant circuits that are the cause of atrial flutter and the re-entrant forms of supraventricular tachycardia. Other rarer congenital causes of monomorphic VT include right ventricular dysplasia, and right and left ventricular outflow tract VT. Polymorphic ventricular tachycardia, on the other hand, is most ...
Objectives: Patients with postural tachycardia syndrome (POTS) often appear anxious and report inattention. Patients with POTS were formally assessed for psychiatric disorders and inattention and compared with patients with attention deficit hyperactivity disorder (ADHD) and control subjects.. Methods: Patients with POTS (n = 21), ADHD (n = 18) and normal control subjects (n = 20) were assessed for DSM-IV psychiatric disorders and completed a battery of questionnaires that assessed depression, anxiety and ADHD characteristics.. Results: Patients with POTS did not have an increased prevalence of major depression or anxiety disorders, including panic disorder, compared with the general population. Patients with POTS had mild depression. They scored as moderately anxious on the Beck Anxiety Inventory but did not exhibit a high level of anxiety sensitivity. Patients with POTS scored significantly higher on inattention and ADHD subscales than control subjects. These symptoms were not present during ...