MERS is caused by a virus called Middle East Respiratory Syndrome Coronavirus (MERS-CoV), which belongs to the Coronavirus family of viruses. MERS Virus belongs to the genus Betacoronavirus. Coronavirus family of viruses includes viruses that usually cause respiratory tract illnesses like the common cold in human beings, and also, these viruses cause a number of diseases in animals. Severe Acute Respiratory Syndrome Coronavirus (SARS-CoV) causes Severe Acute Respiratory Syndrome (SARS).. Continue reading → ...
Compared with Severe Acute Respiratory Syndrome (SARS) and Middle East Respiratory Syndrome (MERS), Corona Virus Disease 2019(COVID-19) spread more rapidly and widely. The population was generally susceptible. However, reports on pregnant women infected with SARS-CoV-2 were very limited. By sharing the clinical characteristics, treatments and outcomes of 18 patients with COVID-19 during late pregnancy, we hope to provide some references for obstetric treatment and management. A total of 18 patients with COVID-19 treated at Renmin Hospital of Wuhan University were collected. The epidemiological characteristics, clinical manifestations, laboratory tests, chest CT and pregnancy outcomes were performed for analysis. 1. 18 cases of late pregnancy infected with SARS-CoV-2 pneumonia were delivered at 35 + 5 weeks to 41 weeks. According to the clinical classification of COVID-19, 1 case was mild type, 16 cases were ordinary type, and 1 case was severe type. 2. According to imaging examinations: 15 (83%) cases
Severe acute respiratory syndrome (SARS) is an emerging infectious disease caused by a novel coronavirus (SARS-CoV). The major clinical features of SARS include fever, dyspnea, lymphopenia, and a rapid progression of pulmonary infiltrates on chest radiologic images. The SARS-related deaths have resulted mainly from pulmonary complications, including progressive respiratory failure due to alveolar damage and acute respiratory distress syndrome (ARDS). Pathological changes in SARS suggest that SARS sequelae such as infiltration of PMN in lung tissue, multiple organ dysfunction and ARDS have been associated with cytokines and chemokine dysregulation. Some patients still manifested lung injury at a time when the viral load was falling also supports the immune nature of the lung damage. We therefore undertook an analysis of dynamic production of cytokine/chemokines in SARS patients with an initial normal chest radiograph in order to improve understanding of disease pathogenesis and improve patient ...
Severe acute respiratory syndrome (SARS) poses a major threat to the health of people worldwide. We performed a retrospective case series analysis to assess clinical outcome and identify pretreatment prognostic correlates of SARS, managed under a standardized treatment protocol. We studied 127 male and 196 female patients with a mean age of 41±14 (range 18-83). All patients, except two, received ribavirin and steroid combination therapy. In 115 (36%) patients, the course of disease was limited. Pneumonitis progressed rapidly in the remaining patients. Sixty-seven (21%) patients required intensive care, and 42 (13%) required ventilator support. Advanced age, high admission neutrophil count, and high initial lactate dehydrogenase level were independent correlates of an adverse clinical outcome. SARS-associated coronavirus caused severe illnesses in most patients, despite early treatment with ribavirin and steroid. This study has identified three independent pretreatment prognostic correlates.
The shedding of severe acute respiratory syndrome coronavirus (SARS-CoV) into saliva droplets plays a critical role in viral transmission. The source of high viral loads in saliva, however, remains elusive. Here we investigate the early target cells of infection in the entire array of respiratory tissues in Chinese macaques after intranasal inoculations with a single-cycle pseudotyped virus and a pathogenic SARS-CoV. We found that angiotensin-converting enzyme 2-positive (ACE2 +) cells were widely distributed in the upper respiratory tract, and ACE2 + epithelial cells lining salivary gland ducts were the early target cells productively infected. Our findings also have implications for SARS-CoV early diagnosis and prevention. Copyright © 2011, American Society for Microbiology. All Rights Reserved ...
Severe acute respiratory syndrome coronavirus (SARS-CoV) causes a respiratory disease leading to death in 10% of the infected people. A mouse adapted SARS-CoV lacking the envelope (E) protein (rSARS-CoV-MA15-ΔE) is attenuated in vivo. To identify E protein domains and host responses that contribute to rSARS-CoV-MA15-ΔE attenuation, several mutants (rSARS-CoV-MA15-E*) containing point mutations or deletions in the amino-terminal or the carboxy-terminal regions of E protein, respectively, were generated. Amino acid substitutions in the amino terminus, or deletion of domains in the internal carboxy terminal region of E protein led to viral attenuation. Attenuated viruses induced minimal lung injury and limited neutrophil influx to the lungs but, interestingly, increased CD4+ and CD8+ T cell counts in BALB/c mice. To analyze the host responses leading to rSARS-CoV-MA15-E* attenuation, the differential gene expression elicited by the native virus and the mutant ones in infected cells was analyzed. The
Severe acute respiratory syndrome (SARS) [1] was an atypical pneumonia.[2] It started in November 2002 in Guangdong Province, in the city of Foshan, of the Peoples Republic of China. The disease was caused by the SARS coronavirus (SARS CoV), a new coronavirus. It was also a part-time STD, it can be spread through both sexual and casual contact. SARS was first reported in Asia in February 2003. Over the next few months, the illness spread to more than 24 countries in Asia, North America, South America, and Europe before the SARS global outbreak of 2003 was contained. According to the World Health Organization (WHO), a total of 8098 people worldwide became sick with SARS during the 2003 outbreak; 774 of these died. After the Chinese government suppressed news of the SARS outbreak, the disease spread rapidly, reaching Hong Kong and Vietnam in late February 2003, and then to other countries via international travellers. The last case in this outbreak occurred in June 2003. There were a total of ...
Taylor JK, Coleman CM, Postel S, Sisk JM, Bernbaum JG, Venkataraman T, Sundberg EJ, Frieman MB. Severe Acute Respiratory Syndrome Coronavirus ORF7a Inhibits Bone Marrow Stromal Antigen 2 Virion Tethering through a Novel Mechanism of Glycosylation Interference. J Virol. 2015 Dec; 89(23):11820-33 ...
Severe acute respiratory syndrome (SARS) is the first emergent and highly transmissible viral disease to appear during the twenty-first century. Patients with SARS develop flu-like fever, headache, malaise, dry cough and other breathing difficulties. Many patients develop pneumonia, and in 5-10% of cases, the pneumonia and other complications are severe enough to cause death. SARS is caused by a virus that is transmitted usually from person to person-predominantly by the aerosolized droplets of virus infected material.. SARS cases provided a test of recent reforms in International Health Regulations designed to increase surveillance and reporting of infectious diseases-and to enhance cooperation in preventing the international spread of disease. Although not an act of bioterrorism, because the very same epidemiologic principles and isolation protocols might be used to both initially determine and initially respond to an act of bioterrorism, intelligence and public heath officials closely ...
Dear Reader,. Thanks for the props! And glad to be of service.. SARS is a serious health concern, and it can be dangerous if you come into contact with it. However, you dont necessarily need to be concerned. The media grasped onto the sensationalized story of SARS because of its rapid spread made possible by the relatively new phenomenon of extensive and frequent world travel and the fast rate of transmission. Luckily the virus was quickly isolated and contained only months after its initial out-break.. SARS, which is an acronym for Severe Acute Respiratory Syndrome, is a contagious and sometimes fatal respiratory illness that is actually a severe form of pneumonia. It is caused by a strain of the coronavirus, which is also the family of viruses that causes the common cold. SARS first appeared in China in 2002, and within six weeks had spread worldwide. Within a month and a half, 8,000 people were infected and 800 had died. SARS dramatically taught us how rapidly world travel can spread ...
Severe acute respiratory syndrome (SARS-CoV-2) due to novel Coronavirus (2019-nCoV) related infection (COVID-19) is characterized by severe ventilation perfusion mismatch leading to refractory hypoxemia. To date, there is no specific treatment available for 2019-nCoV. Nitric oxide is a selective pulmonary vasodilator gas used as a rescue therapy in refractory hypoxemia due to acute respiratory distress syndrome (ARDS). In has also shown in-vitro and clinical evidence that inhaled nitric oxide gas (iNO) has antiviral activity against other strains of coronavirus.. The primary aim of this study is to determine whether inhaled NO improves oxygenation in patients with hypoxic SARS-CoV2.. This is a multicenter randomized controlled trial with 1:1 individual allocation. Patients will be blinded to the treatment.. Intubated patients admitted to the intensive care unit with confirmed SARS-CoV-2 infection and severe hypoxemia will be randomized to receive inhalation of NO (treatment group) or not ...
Severe acute respiratory syndrome (SARS) is a highly contagious, serious and potentially life-threatening form of pneumonia. (SARS (Severe syndrome))
On March 21, 2003, this report was posted on the MMWR website (http://www.cdc.gov/mmwr). Severe acute respiratory syndrome (SARS) is a condition of unknown etiology that has been described in patients in Asia, North America, and Europe. This report summarizes the clinical description of patients with SARS based on information collected since mid-February 2003 by the World Health Organization (WHO), Health Canada, and CDC in collaboration with health authorities and clinicians in Hong Kong, Taiwan, Bangkok, Singapore, the United Kingdom, Slovenia, Canada, and the United States. This information is preliminary and limited by the broad and necessarily nonspecific case definition. As of March 21, 2003, the majority of patients identified as having SARS have been adults aged 25--70 years who were previously healthy. Few suspected cases of SARS have been reported among children aged ,15 years. The incubation period for SARS is typically 2--7 days; however, isolated reports have suggested an incubation ...
The WHO Global Conference on Severe Acute Respiratory Syndrome (SARS), was held in the Sunway Lagoon Resort Hotel, Persiaran Lagoon, Bandar Sunway, 46150, Petaling Jaya, Selangor Darul Ehsan, Kuala Lumpur, Malaysia on 17-18 June 2003.. ...
Liu, E.H., Koh, K.-F., Chen, F.-G. (2004-06). Outbreak of severe acute respiratory syndrome in Singapore and modifications in the anesthesia service [13]. Anesthesiology 100 (6) : 1629-1630. [email protected] Repository ...
Liu, E.H.,Koh, K.-F.,Chen, F.-G. (2004). Outbreak of severe acute respiratory syndrome in Singapore and modifications in the anesthesia service [13]. Anesthesiology 100 (6) : 1629-1630. [email protected] Repository ...
Negative public reactions to emerging infectious diseases can adversely affect population health. We assessed whether social support was associated with knowledge of, worry about, and attitudes towards AIDS and severe acute respiratory syndrome. Our findings suggest that social support may be central to our understanding of public responses to emerging infectious diseases.
The global outbreak caused by the severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) has been declared a pandemic by the WHO. As the number of imported SARS-CoV-2 cases is on the rise in Brazil, we use incidence and historical air travel data to estimate the most important routes of importation into the country.
While Toronto recovers from the lingering effects of its Severe Acute Respiratory Syndrome outbreak, health experts are worried the city is poised for an ...
Despite difficult challenges during responses to the terrorist attacks of September 11, 2001, Hurricane Katrina, and the 2009 Pandemic Influenza A/H1N1 and severe acute respiratory syndrome outbreaks, no North American emergency to date has overwhelmed intensive care unit (ICU) services on a widespread basis since the modern development of the field of critical care. However, planners have recognized that in a future public health emergency we may not be so fortunate. To deal with very large emergencies involving many patients whose survival depends on immediate access to intensive care, an international Task Force for Mass Critical Care proposed recommendations in January 2007 to extend critical care resources for the adult population, referred to as the Emergency Mass Critical Care (EMCC) approach (1-5).. The EMCC approach triples critical care capabilities for a period of up to 10 days in a very large public health emergency by focusing on immediately life-saving interventions, while delaying ...
SARS is mainly spread by close person-to-person contact. After two to seven days, SARS patients may develop a dry cough with most developing pneumonia.
this news reminded me a bit different thing. recently Japanese TV reported that theres growing number of pulmonary edema like symptom spreading widely in Japan, caused by spraying anti-rain coating chemical such as ScotchGuard in closed air space like inside of cars or houses. people here often use such chemicals spray on skiwears and shoes before going to ski, then get sudden breathing problem during skiing and then hospitalized. if theres no virus or bacterium found on this SARS case, Im curious to other type of chemicals in relation. BTW you can even make vapor that causes temporary breathing problems by just burning red pepper in oil. dont try this at home. (I accidentally did while cooking curry. it was horrible.). ...
The number of SARS cases, and the number of countries reporting such cases, continues to increase worldwide. Transmission within hospitals and households continues in some areas, and transmission within communities (e.g., Hong Kong) continues to be reported. In the absence of a complete understanding of how SARS is transmitted, efforts to limit transmission in the United States have focused on early identification of potential cases through surveillance and implementation of infection-control measures in health-care settings and the community. CDC has developed interim infection-control guidelines for use in U.S. health-care and household settings (8). These recommendations are based on experience in the United States to date and will be revised as more information becomes available. Infection-control practitioners and clinicians providing medical care for patients with suspected SARS should consult these guidelines frequently to keep current with recommendations. Transmission in health-care ...
In the present study we investigated, for the first time, the susceptibility of primary human Mφ and DC and their subsequent responses to SARS-CoV infection by focusing on their phenotypic expression, secretion of inflammatory cytokines, phagocytic activity, and T cell-priming capacity. The findings of our studies are: 1) SARS-CoV could not productively infect Mφ and DC (Figs. 1⇑ and 2⇑); 2) incubation with SARS-CoV did not add to the spontaneous cell death of primary Mφ and DC; on the contrary, it enhanced their viability (Fig. 3⇑) independently of the presence of exogenous cytokines; 3) exposure to live SARS-CoV exerted a diverse effect on the biology of DC vs Mφ, in which it up-regulated the expression of MHC class II, CD40, CD83, and CD86, on the surface of DC but not Mφ. Consequently, it also greatly enhanced DC ability to stimulate the proliferation of allogeneic T cells (Figs. 4⇑ and 8⇑). In contrast, it impaired the phagocytic activity of Mφ (Fig. 7⇑); and 4) not only ...
Using the purified proteins as antigens in ELISA assays for antibodies in the sera of SARS patients we found that the assay using truncated S-N fusion protein has a clearly higher sensitivity than those using intact N protein or truncated S and N proteins, and virtually as high as the assay using whole SARSCoV lysate (Table 2). The results indicated the N and S protein were complementary in detecting SARS-specific antibodies. This is consistent with previous studies [28, 29]. Five positive sera to SARSCoV lysate antigen were all tested positive against SARSCoV N protein but negative against SARSCoV truncated N-S protein. These sera were also tested positive against N proteins of HCoV 229E and HCoV OC43 (data not shown), which could be reasonably explained partly by existence of other HCoV infections in these humans. The truncated S-N fusion protein was also subjected to ELISA with mice antisera against SARSCoV (BJ01), HCoV 229E and HCoV OC43. Only mice antiserum against SARSCoV(BJ01) tests ...
Severe acute respiratory syndrome (SARS) originated in southern China in November 2002, and soon spread to people on six continents, killing nearly 800 people all told. For the first time, researchers have identified a crucial SARS receptor in vivo that helps explain how and why SARS infection causes lung failure. The findings should help researchers treat other diseases that compromise lung function. Previous research using cell lines had identified ACE2, a protein involved in regulating blood pressure, as a potential SARS receptor. Now Josef Penninger of the Institute of Molecular Biotechnology (IMBA) in Vienna, Austria, and his colleagues have tested the hypothesis in mice. In a paper published online yesterday by Nature Medicine, they report that SARS infection, and specifically the SARS protein known as Spike, reduces ACE2 expression. As a result, blood vessels in the lungs become damaged and the lung becomes flooded as a result. In a second paper published in the current issue of the ...
SARS is a condition associated with substantial morbidity and mortality. It appears to be of viral origin, with patterns suggesting droplet or contact transmission. The role of human metapneumovirus, a novel coronavirus, or both requires further investigation.
The risk factors for SARS are close contact within the previous 10 days with a probable case of SARS, recent travel within the previous 10 days to a reported affected area.
The lungs and gastrointestinal tract have been demonstrated to be the only major organ systems that support SARS-CoV replication. After establishment of infection, SARS-CoV causes tissue damage by (1) direct lytic effects on host cells and (2) indirect consequences resulting from the host immune response.
Fourteen cytokines or chemokines were analyzed on 88 RT-PCR-confirmed severe acute respiratory syndrome (SARS) patients. IFN-gamma, IL-18, TGF-beta, IL-6, IP-10, MCP-1, MIG, and IL-8, but not of TNF-alpha, IL-2, IL-4, IL-10, IL-13, or TNFRI, were highly elevated in the acute phase sera of Taiwan SAR …
The Chinese government is beginning to show signs that it is turning its attention to fighting HIV/AIDS now that the last 12 patients with severe acute ...
This study aimed to determine the short term outcome and risk factors on presentation that were associated with an adverse clinical course and mortality in a group of predominantly Chinese patients with SARS. Age above 60 and the presence of comorbid conditions were associated with a higher rate of mortality. The presence of diabetes mellitus and/or cardiac diseases was related both to a higher mortality rate and an adverse outcome. The crude mortality rate of 15.7% and 21 day mortality rate of 10% (standard error 3%) appeared higher than in other recently published studies5-7 but, as pointed out by Donnelly et al,11 it is difficult to calculate the true mortality rate of the disease while the epidemic is still continuing and it is impossible to ascertain which of the remaining patients will eventually die or be discharged. The estimated case fatality rate calculated by Donnelly et al was 13.2% for patients younger than 60 and more than 40% for those aged over 60.11. The presence of ...
In indented style, main headings are followed by indented subheadings, each on its own line. In run-in style, subheadings appear continuously, not on separate lines, and are separated by commas.Indented: SARS (severe acute respiratory syndrome), 75 79, 145 148 in China, 187 189 drug therapy for antibiotics, 18, 20 corticosteroids, 357 interferon alfa, 402 etiology of, 93, 105, 117 quarantine for, 167, 235 in Toronto, 280 Run-in: SARS (severe acute respiratory syndrome), 75 79, 145 148, in China, 187 189, drug therapy for, 18, 20, 357, 402, etiology of, 93, 105, 117, quarantine for, 167, 235, in Toronto, 280 The
In indented style, main headings are followed by indented subheadings, each on its own line. In run-in style, subheadings appear continuously, not on separate lines, and are separated by commas.Indented: SARS (severe acute respiratory syndrome), 75 79, 145 148 in China, 187 189 drug therapy for antibiotics, 18, 20 corticosteroids, 357 interferon alfa, 402 etiology of, 93, 105, 117 quarantine for, 167, 235 in Toronto, 280 Run-in: SARS (severe acute respiratory syndrome), 75 79, 145 148, in China, 187 189, drug therapy for, 18, 20, 357, 402, etiology of, 93, 105, 117, quarantine for, 167, 235, in Toronto, 280 The
The first case of severe acute respiratory syndrome (SARS) in France was diagnosed in March 2003. We conducted a serological survey to assess whether or not asymptomatic persons who had been in contact with this patient during his infectious stage had been infected. They were interviewed and asked to provide a blood sample for SARS coronavirus immunoglobulin G antibody testing. Despite the likely high infectivity of the SARS patient, no asymptomatic SARS infection was found in any of the 37 contacts included. These findings support a SARS case definition that is essentially based on clinical and epidemiological assessment, should SARS re-emerge.
3St. Christophers Hospital for Children, Department of Pediatrics, Philadelphia, USA DOI : 10.35333/jrp.2020.215 Severe acute respiratory syndrome corona virus 2 (SARS-CoV-2) which is the human coronavirus and a member of the Coronaviridae family leads to fatal pneumonia cases. Severe acute respiratory syndrome corona virus 2 attaches to the cells in the human body through binding to the angiotensin converting enzyme 2 (ACE2) receptor with the spike (S) protein. Firstly, SARS-CoV-2 arised in China in late 2019 and was reported to the World Health Organization (WHO). The World Health Organization named the disease caused by this virus as corona virus disease (COVID)-19. SARS-CoV-2 which has human-to-human transmission through droplets, direct contact and aerosol routes have affected more than 10 million people and caused more than 500 thousand deaths. Clinical symptoms of COVID-19 are fever, dry cough, sore throat, respiratory distress, lung damage, and diarrhea. In severe cases, mechanical ...
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Current treatment schemes for severe acute respiratory syndrome (SARS) include broad-spectrum antibiotics, glucocorticoids, and ribavirin. We evaluated the susceptibility of the SARS-related coronavirus (SARS CoV) to ribavirin and interferon (IFN)- alpha in vitro by use of cytopathic effect, plaque assay, and immunoblot analysis. Ribavirin did not inhibit viral growth at concentrations attainable in human serum. In contrast, IFN- alpha showed an in vitro inhibitory effect starting at concentrations of 1000 IU/mL. In conclusion, ribavirin alone is unlikely to be beneficial in the prophylaxis or treatment of SARS CoV infections. Clinical trials with IFN- alpha might be justified to determine a beneficial effect on the outcome of SARS ...
In 2004, I did some reportage for the Royal Society from their meeting on emerging viral infections. The meeting was held just after the worldwide SARS outbreak that threw nations into chaos and had the more susceptible parts of the media hyping the end of the world. Of course, SARS, an emerging pathogen, was lethal and had devastating effects on thousands of people.. Ultimately, the first SARS outbreak was controlled, and a subsequent epidemic is yet to emerge. Severe acute respiratory syndrome, the disease caused by a highly infectious RNA coronavirus, remains in waiting. SARS is still an issue, it can, when required, undergo frequent mutations, which adds unpredictability to a future outbreak. There is no vaccine, assay, or treatment yet. Health officials can only resort to isolation and quarantine to control its spread.. In the meantime, scare stories surrounding the potential for avian influenza H5N1 have filled many column inches and web estate since that strain was first identified. We ...
A patient with a suspected case of severe acute respiratory syndrome, or SARS, was treated Wednesday afternoon at Gloucester Convenient Care, health officials said.The suspected case, under
The worldwide outbreak of severe acute respiratory syndrome (SARS) has had its epicenter in Asia. Lee and colleagues from the Prince of Wales Hospital in Hong Kong described pooled data on clinical presentations and outcomes from their cluster of patients with SARS.. In early March, an outbreak of atypical, severe pneumonia was recognized, and a special multidisciplinary team was mobilized. Before the causative agent (a novel coronavirus) was identified, the team used the SARS case criteria established by the Centers for Disease Control and Prevention: temperature greater than 38°C (100.4°F), chest radiography with consolidation, and contact with a previously identified patient with SARS. The authors derived their information from a group of 138 infected persons, 50 percent of whom were health care workers secondarily infected by index patients with SARS.. Initial treatment included empiric antibiotics, followed by corticosteroids and ribavirin when fever persisted for more than 48 hours and ...
Severe acute respiratory syndrome (SARS) is a dangerous infection with pandemic potential. It emerged in 2002 and its aetiological agent, the SARS Coronavirus (SARS-CoV), crossed the species barrier to infect humans, showing high morbidity and mortality rates. No vaccines are currently licensed for SARS-CoV and important efforts have been performed during the first outbreak to develop diagnostic tools. Here we demonstrate the transient expression in Nicotiana benthamiana of two important antigenic determinants of the SARS-CoV, the nucleocapsid protein (N) and the membrane protein (M) using a virus-derived vector or agro-infiltration, respectively. For the M protein, this is the first description of production in plants, while for plant-derived N protein we demonstrate that it is recognized by sera of patients from the SARS outbreak in Hong Kong in 2003. The availability of recombinant N and M proteins from plants opens the way to further evaluation of their potential utility for the development of
Two new cases of severe acute respiratory syndrome were reported in Connecticut Tuesday, both involving people who recently traveled in the Far East.The two patients are recovering, according to
(CIDRAP News) Five SARS (severe acute respiratory syndrome) patients have been released from Chinese hospitals this week, leaving only two still hospitalized with the disease, according to reports from China.
The virus has been identified as 2019-nCoV, which stands for a novel coronavirus discovered in 2019. According to the World Health Organization, it is from the same family of viruses as Severe Acute Respiratory Syndrome (SARS-CoV), but it is not the same virus. In February, the WHO gave the disease caused by this virus a new name: COVID-19. Coronaviruses are a large family of viruses, most of which are harmless for humans. Four types are known to cause colds while two other types can cause severe lung infections-SARS and Middle Eastern Respiratory Syndrome (MERS), both of which are similar to COVID-19 ...
In their article, HHS/CDC Legal Response to Outbreak of Severe Acute Respiratory Syndrome (SARS), Misrahi, et al. (Emerg Infect Dis 2004:10:353-5.) describe the
General Information. The outbreak of severe respiratory illness related to the new coronavirus (COVID-19) continues to have an expanding impact internationally. The World Health Organization (WHO) provides regular updates which guide our recommendations. The latest WHO statements can be found at WHO WEBSITE ON CORONAVIRUS DISEASE (COVID-19) OUTBREAK.. COVID-19 is a virus in the coronavirus family. Coronaviruses in this family are responsible for illnesses that range from the common cold to more serious illnesses such as Severe Acute Respiratory Syndrome (SARS-CoV) and Middle Eastern Respiratory Syndrome (MERS-CoV). COVID-19 is a new virus and so health officials are still learning about its impact and severity. At this time, it appears to cause an illness similar to the flu with the most common signs of infection being fever, cough and shortness of breath. In severe cases patients can develop pneumonia, severe respiratory distress, kidney failure and death.. Prevention. Recommendations for ...