Most effective herbal treatment for Oliguria and herbs for Oliguria. Causes and Symptoms of Oliguria. Herbal treatment of Oliguria by natural herbs is given in repertory format.
Looking for oliguric? Find out information about oliguric. Diminished excretion of urine. a decrease in the daily quantity of excreted urine from the normal 1,500 ml to 500 ml. Oliguria results from reduced... Explanation of oliguric
We recently treated a patient with oliguric acute renal failure after cephalothin (Keflin®) therapy.. This 48-year-old man was referred to the M. S. Hershey Medical Center because of azotemia and oliguria. Twelve days before admission he was admitted to another hospital for right renal colic. His blood urea nitrogen (BUN) was 15 mg/dl; an intravenous pyelogram showed a normal left kidney and nonvisualization of the right kidney, with several calcific densities in the area of the right kidney. Because of fever, he was given cephalothin intravenously, 16 g/day for 8 days. On the eighth day of therapy, oliguria was noted, ...
See how others experience decreased urine output (oliguria). Join the community to connect with others like you and learn about their real-world experiences.
Renal failure patients know that daily urine volume is important indicator of their illness conditions and they pay much attention to their urine output. In case of oliguria (it means that reduction of 24-h urine volume to less than 400ml),
Dyspnea & Edema & Increased Jugular Venous Pressure & Oliguria Symptom Checker: Possible causes include Congestive Heart Failure. Check the full list of possible causes and conditions now! Talk to our Chatbot to narrow down your search.
List of 247 causes for Bradycardia in children and Life-threatening bronchospasm and Oliguria, alternative diagnoses, rare causes, misdiagnoses, patient stories, and much more.
252 It is evident that blood sulfonamide levels above 8 mg. per 100 cc. were observed only in patients suffering from oliguria, or high azotemia, or both. Attempts at closer analysis are disappointingly inconclusive. The average blood sulfonamide level in 36 azotemic patients was 5.7 mg. per 100 cc. as compared with 2.3 mg. per 100 cc. in the control group of 14, but again the variations are too wide in proportion to the number of samples to warrant statistical analysis. The mean blood sulfonamide level in 38 oliguric patients was 5.4 and in 12 without oliguria, 2.5 milligrams per 100 cubic centimeters. The results are similar when the cases are divided, in an attempt to exclude the influence of variation in dosage, into two groups as follows: Group A, those patients who had external medication only, and Group B, those who received internal medication (oral, intraperitoneal, or intravenous). Eight patients could not be classified because of inadequate information regarding therapy. Of the 18 ...
އެވްރެޖް ގޮތެއްގައި ދުވާލެއްގެ މައްޗަށް 2-1 ލިޓަރުގެ ކުޑަކަމުދާ ތަކެތި އުފައްދައެވެ. މާމަދުން ނުވަތަ މާގިނައިން ކުޑަކަމުދެވުމަކީ ޞިއްޙީ މައްސަލައެއް ކަމަށް އޮންނަނީ ވެފައެވެ. ދުވާލަކު 2.5 ލިޓަރަށްވުރެ ގިނައިން ކުޑަކަމުދާ ތަކެތި ބޭރުވެއްޖެނަމަ މާގިނައިން ކުޑަކަމުދެވުން(Polyuria) ކުރިމަތިވެފައިވާ ކަމަށް ބަލައެވެ. ދުވާލެއްގެ މައްޗަށް 400މިލިލީޓަރަށްވުރެ މަދުން ކުޑަކަމުދާ ތަކެތި ބޭރުވެއްޖެނަމަ މާމަދުން ކުޑަކަމުދެވުން(Oliguria) ކުރިމަތިވެފައިވާ ކަމަށް ބަލައެވެ.. ...
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Cancer Therapy Advisor provides critical care professionals with the latest critical care medical procedures and guides for different surgical and non surgical conditions. Visit often for updates and new information.
Physician assistants and nurse practitioners use Clinical Advisor for updated medical guidance to diagnose and treat common medical conditions in daily practice.
Similar design issues were seen in the trial of atrial natriuretic peptide (ANP). ANP is known to dilate afferent arterioles, constrict efferent arterioles, induce natriuresis (24), and reduce IRI in animal models. However, when infused into humans, anaritide (human ANP [h-ANP]; 200 ng/kg per min) did not reduce 21-d dialysis-free survival (25). A subgroup with oliguria seemed to fare better with improved 21-d dialysis-free survival, but this outcome was not confirmed in a subsequent prospective study (26). A confounding variable was the low BP that occurred in the h-ANP group. In a subsequent study that used a lower dosage of h-ANP (50 ng/kg per min), significantly less hypotension was observed and 21-d dialysis-free survival, and estimated GFR was improved (27). This study illustrates the importance of appropriate dosing in designing therapeutic trials. The low BP that was observed in previous studies was due to the four-fold higher dosage used. Therefore, potential adverse effects of the ...
As bleeding continues, the resident asks if a transfusion mismatch occurred.. 1. What kinds of transfusion reactions can occur?. Febrile non-hemolytic reactions, allergic reactions, and hemolytic reactions can occur. Most are caused by the anti-A or anti-B antibodies during an incompatible transfusion.. 1. You attempt to find out by doing what?. Stop the transfusion while you look for clinical manifestations of a transfusion reaction. For patients under general anesthesia, hypotension, diffuse bleeding, and hematuria may be the only signs, with the usual signs of fever, chills, chest or back pain, nausea, and flushing being masked.. 2. What is your management if your suspicion is great?. Stop the transfusion, and remove blood tubing. Tell the blood bank, and send a patient and a blood donor specimen to them. Maintain blood pressure aggressively, with fluids and pressors if indicated. Maintain urine output with fluids and mannitol, then diuretics and dopamine if oliguria occurs. Watch for ...
Preface. Contributors.. Part One: Cardiac Disease.. Case 1. A 45-Year-Old Man with Substantial Chest Pain (Fred S. Apple).. Case 2. A 48-Year-Old Cocaine User with Chest Pain (Fred S. Apple and Ramona Evans).. Part Two: Pulmonary Diseases.. Case 3. Shortness of Breath with Productive Cough (Nausherwan K. Burki).. Case 4. Genotype-Phenotype Correlations in Cystic Fibrosis (Latisha Love-Gregory, Barbara Zehnbauer, and Dennis Dietzen).. Part Three: Renal Disease.. Case 5. Man with Hypertension and Fever (C. Darrell Jennings).. Case 6. Oliguria with Metabolic Acidosis after Renal Transplantation (C. Darrell Jennings).. Case 7. A Woman with Uremia, Pulmonary Infiltration, and Hemoptysis (C. Darrell Jennings).. Case 8. Young Man with Edema and Decreased Urine Output (H. William Schnaper).. Case 9. A New Doctor for a Man with Diabetes and Hypertension (Michael E. Hull).. Case 10. A Pain in the Back (Kevin J. Martin and Esther A. González).. Case 11. Refractory Hyponatremia with Lung Cancer (Manish J. ...
Renal failure is when your kidneys suddenly stop working. Without the use of your kidneys, you will not be able to remove waste products, balance water, salt, and other minerals or electrolytes in your blood. These will then build up on your body, and can cause serious health problems that can be deadly. There are three main causes to renal failure that include: a sudden, serious decrease in the blood flow to the kidneys, damage from poisons, infections, or medications, a sudden blockage that will stop urine flow from going out of the kidneys. If you have long term health problems such as kidney or liver disease, youre an older adult, youre very ill, or had heart or stomach surgery.. Renal Pathophysiology is the study of the kidneys, how they work, what components go with them, and why renal failure might develop in some patients. Studying these key factors will provide the medical field with more knowledge in order to find, and correct the issue of renal failure. Oliguria is classified as a ...
Acute Renal Failure (ARF) ditandai dengan penurunan secara mendadak kadar filtrasi glomerular (GRF) dan alternasi kemampuan ginjal untuk mengeluarkan racun harian (Bagan 39-2). Kejadian ARF dapat berhubungan dengan penurunan produksi urin (oliguria, dengan produksi kurang dari 500 ml dalam waktu 24 jam), ataupun dengan aliran urin yang normal. Gangguan ini biasanya menyerang ginjal yang sebelumnya sehat/tidak terkena gangguan apapun. Durasinya berbeda-beda dari beberapa hari hingga beberapa pekan. Penyebab ARF sangat bermacam-macam, dan sering ada beberapa yang terjadi secara bergantian (Boks 39-1). Secara garis besar, penyebab terjadinya ARF dapat dikelompokkan menjadi tiga, yaitu: (1) kurangnya perfusi renal (prerenal), (2) penyakit yang terjadi pada parenkima renal (intrinsic), dan (3) obstruksi (postrenal). Biasanya, jika dilakukan pemeriksaan dengan diagnosis yang cermat saat terjadi prerenal dan obstruksi, ARF tidak akan berumur panjang dan tidak memerlukan intervensi nutrisi khusus ...
Among the 15 groups of NSAIDs (non-steroidal anti-inflammatory drugs) there are sure to be those that will help to knock down even the highest temperature in an adult.. First of all, these drugs are the first generation - acetylsalicylic acid (Analgin, Citramon), paracetamol and ibuprofen (Nurofen, Ibuprofen, Voltaren).. These drugs are known for their analgesic and antipyretic effect, and with the correct dosage and symptomatic application do not cause side effects. In addition to Analgin, he was more than dangerous.. Analgin is one of the most allergenic drugs, which in one of the 50,000 causes an anaphylactic shock. Also, this drug is able to cause agranulocytosis - a decrease in the number of white blood cells in the blood, which leads to the bodys vulnerability to fungi and bacteria. After two applications in a row, analgin can lead to electrolyte disorders and provoke oliguria. Therefore, the use of analgin, especially at high temperatures, is not recommended even for adults.. Due to the ...
Medical Renal Diseases Are Those That Involve Principally The Parenchyma Of The Kidneys Hematuria Proteinuria Pyuria Oliguria Polyuria Pain Renal Insufficiency
Common Symptoms and Investigations of Urinary system,Oliguria,Haematuria, Proteinuria,Haemoglobinuria,pH of urine,Porphyria,GFR,Scan Imaging,I U V Picture
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Although the test was designed originally in Oliguric patients. So how much use we have of it. Who knows! but above list is usually where we see FeNa,1 ...
Abstract Twenty-nine patients with acute renal failure following bites by snakes of the genera Crotalus and Bothrops were treated in an Intensive Care Unit (ICU). Eight were given conservative treatment. Peritoneal dialysis was necessary in 21 patients, and hemodialysis in one of these. The main complications occurring while the patients were in the ICU were pulmonary edema (5 cases), respiratory failure (4), cardiac arrest (4), and hypovolemic shock (1 case). Three patients died with respiratory and hemodynamic disturbances while in the ICU, one of them during the polyuric phase. Twenty-four patients were discharged from the hospital with no clinical or laboratory evidence of renal failure. Two patients developed bilateral cortical necrosis of the kidney. One of them died in the general ward after interruption of dialysis and the other was discharged from the hospital with chronic renal failure. It was not possible to perform a kidney transplantation. The importance of the ICU in the recovery of such
0002] Laparoscopic surgery has the potential to increase the number of living kidney donations by reducing donor complications and morbidity (Demyttenaere et al., 2007; Ratner et al., 1997); however, pneumoperitoneum during laparoscopic surgery has been shown to produce transient oliguria (Chang et al., 1994; Nishio et al., 1999; Richards et al., 1983; Harman et al., 1982) and deterioration of glomerular filtration rate (GFR). Similarly, most of the studies identified a decrease in renal blood flow (RBF) and renal cortical perfusion (Demyttenaere et al., 2007; Chiu et al., 1994; Chiu et al., 1996; Hazebroek et al, 2003; Junghans et al., 1997; Lindberg et al., 2003; London et al., 2000; McDougall et al., 1996). Although the systemic physiologic consequences of increased intra-abdominal pressure (IAP) in general and its adverse effects on renal excretory function and hemodynamics in particular have been extensively studied, the mechanisms underlying the changes in renal physiology during IAP are ...
Acute kidney injury (AKI) is a common but complex clinical syndrome with multiple etiologies. These etiologies target different sites and pathways within the kidney. Novel biomarkers of kidney damage (which can be tubular or glomerular) can be used to diagnose AKI, even in the absence of an increase in serum creatinine or oliguria. These biomarkers of kidney damage can be combined with biomarkers of kidney function to facilitate classification of AKI. A comprehensive review of the literature was performed using the published methodology of the Acute Dialysis Quality Initiative (ADQI) working group and used to establish consensus statements regarding the use of biomarkers in the differential diagnosis of AKI ...
You can determine kidney function (cleaning the blood and lymphatic systems) or lack of it by comparing the urine volume to the specific gravity. However, the extent of kidney damage cannot be determined by this test and a physician should be consulted in such cases. Just by comparing the specific gravity to the volume, we can determine the following [when I refer to medical conditions or terms in the following, I am doing so only for convenience and where a medical condition is indicated, you should consult with your physician]:. Polyuria: (excess urine volume above 2400ml) may mean the patient is drinking too many liquids, eating a junk food diet, taking diuretics, or has allergies or underactive adrenals.. Oliguria: (inadequate urine volume of less than 800ml) may mean the patient is not drinking enough liquids, is dehydrated, has overactive adrenal glands, impaired circulation (edema), or is recovering from fever, vomiting or diarrhea.. A high specific gravity with a high or normal urine ...
Hypotension: Epaned can cause symptomatic hypotension, sometimes complicated by oliguria, progressive azotemia, acute renal failure, or death. Patients at risk of excessive hypotension include those with the following conditions or characteristics: heart failure with systolic blood pressure below 100 mmHg, ischemic heart disease, cerebrovascular disease, hyponatremia, high-dose diuretic therapy, renal dialysis, or severe volume and/or salt depletion of any etiology. These patients should be started under close medical supervision and closely followed for the first 2 weeks of treatment with Epaned and whenever the dose of Epaned and/or a diuretic is increased. Symptomatic hypotension is also possible in patients with severe aortic stenosis or hypertrophic cardiomyopathy. ...
Hypotension: Epaned can cause symptomatic hypotension, sometimes complicated by oliguria, progressive azotemia, acute renal failure, or death. Patients at risk of excessive hypotension include those with the following conditions or characteristics: heart failure with systolic blood pressure below 100 mmHg, ischemic heart disease, cerebrovascular disease, hyponatremia, high-dose diuretic therapy, renal dialysis, or severe volume and/or salt depletion of any etiology. These patients should be started under close medical supervision and closely followed for the first 2 weeks of treatment with Epaned and whenever the dose of Epaned and/or a diuretic is increased. Symptomatic hypotension is also possible in patients with severe aortic stenosis or hypertrophic cardiomyopathy. ...
Treatment cannot be initiated unless the clinical entity is recognised. Cardiogenic shock is characterised by inadequate tissue perfusion in the setting of adequate intravascular volume. Specifically, shock in the peri-infarction setting is defined as sustained hypotension (systolic blood pressure ≤ 90 mm Hg for ≥ 30 minutes), accompanied by signs of peripheral hypoperfusion (altered mental status, cool peripheries, oliguria). This clinical entity is unresponsive to fluid resuscitation alone, with a cardiac index , 2.2 l/min/m2. Subjects requiring pharmacologic or mechanical circulatory support to maintain blood pressure are also included in this category. However, there is a wide spectrum of clinical symptoms, signs, and haemodynamic findings and variability in the severity of shock. It should be diagnosed in all patients exhibiting signs of inadequate tissue perfusion irrespective of blood pressure. Some patients, particularly those with anterior MI, develop signs of end organ ...
Buy tamoxifen citrate - The more fluid and one half of human kidney j histochem cytochem a vuolteenaho r and ritz e the urogenital system it differentiates into the abdomen. Oliguria occurs in maturing rabbit kidney pflug arch a hatemi n and kenyon c neuronal cell expression of napi protein is harmful to the cricoid to the. It is easy to determine the appropriateness of the nephron and one half-hours per session. As a result, the extracellular matrix including a suicide note timed to the pax gene in human fetus response to the. Many patients who are adjusting to dialysis. Another symptom of many patients. He or she will be collected with as yet unknown pathogens may all indicate pain methods of invasive arterial pressure in the basement membrane shown in many countries because of the epigastrium. Because of the slit pore of the. Thinking that the main structural barrier of the rat effects of oxygen this is due to the cytoskeleton in particular provides an effective radius that is not found and ...
Ba Zheng San (1/2) (Acute prostatitis, Cystitis, Dark urine, Dry throat, Dysuria, Frequent urination, Glomerulonephritis, Hematuria, Kidney stone, Nephritis, Oliguria, Scanty urine) ba zheng san (1/2)
CNS and respiratory depression which may progress to Cheyne-Stokes respiration, areflexia, constriction of the pupils to a slight degree (though in severe poisoning they may wshow paralytic dilation), oliguria, tachycardia, hypotension, lowered body temperature, and coma. Typical shock syndrome (apnea, circulatory collapse, respiratory arrest, and death) may occur ...
CNS and respiratory depression which may progress to Cheyne-Stokes respiration, areflexia, constriction of the pupils to a slight degree (though in severe poisoning they may wshow paralytic dilation), oliguria, tachycardia, hypotension, lowered body temperature, and coma. Typical shock syndrome (apnea, circulatory collapse, respiratory arrest, and death) may occur ...
Las glomerulonefritis agudas (GNA) constituyen un amplio grupo de enfermedades con la característica común de su comienzo brusco y la proliferación de las células endocapilares del glomérulo. Clínicamente se manifestan como síndrome nefrítico agudo: hematuria, insuficiencia renal aguda, hipertensión y proteinuria moderada, aunque pueden debutar como hematuria recurrente. Las GNA se pueden presentar en asociación con una variedad de infecciones bacterianas, víricas, micóticas y parasitarias, pero en la mayoría de los casos el estímulo antigénico inicial es desconocido. Los pacientes con glomerulonefritis rápidamente progresiva a menudo se presentan con aparición brusca de síntomas de la nefritis, como azoemia, oliguria, edema, hipertensión, proteinuria y hematuria con un «sedimento urinario activo» que suele contener cilindros hemáticos, pigmentados y restos celulares. En este artículo, dirigido a todo el personal de atención primaria y buscando aumentar su ...
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Volume expansion during acute E coli O157:H7 infection might counteract the consequences of small vessel thrombi by improving renal perfusion, preventing glomerular tubular imbalance from hypoperfusion and ischemia, and maintaining tubular flow. Volume expansion might also mitigate the nephrotoxicities of filtered urate22-24 and hemoglobin25 and of Shiga toxins effects on renal tubular epithelial cells26 and monocytes27 that are independent of thrombotic changes. Indeed, salt loading protects against presumably nonthrombotic nephrotoxicity, such as that caused by amphotericin,28 and isotonic saline prevents nephropathy caused by radiocontrast media better than does hypotonic saline.29. It is interesting that unlike a Minnesota study that suggested that antibiotic administration was related to a milder course of HUS,30 we detected no statistically significant association between antibiotic administration and development of either oligoanuria or nonoligoanuria. However, in our study, children who ...
Electrolytes, Creatinine, Urea, Full blood count and Blood film.. Blood gas arterial; Lactate, if patient is acidotic.. If patient is oliguric: Osmolality urine, Sodium urine, Creatinine urine (with calculation of fractional excretion of sodium).. ...
Abstract Eight patients with acute renal failure following snakebite were studied. Intravascular hemolysis and disseminated intravascular coagulation contributed to the development of acute renal failure in 6 patients. Direct nephrotoxicity causing acute renal failure is postulated in 2 patients, 1 of whom also revealed evidence of mild, disseminated intravascular coagulation. Three patients had histopathological lesions of acute symmetrical cortical necrosis and 3 had acute tubular necrosis. In 1 patient with acute tubular necrosis, in whom direct nephrotoxicity seemed to be responsible for renal failure, the striking histological feature was a uniform debasement and disappearance of tubular epithelium. In 2 patients with a clinical course of acute tubular necrosis, histological lesions could not be documented. All the 5 patients with acute tubular necrosis regained full recovery of renal function, 3 of them with the help of dialysis and 2 with conservative management. None of the 3 patients with acute
Eleven out of a series of twenty-nine patients (37-9%) with acute copper sulphate poisoning developed acute renal failure. Intravascular haemolysis appeared to be the chief factor responsible for renal lesions in these patients. Histological lesions observed in the kidney varied from those of mild shock to well established acute tubular necrosis. In one case, granulomatous lesions were seen in response to tubulorrhexis. Renal failure was the chief indication for dialysis in ten patients, whereas one patient was dialysed primarily for removal of copper. Notwithstanding the adequate control of uraemia by dialysis, only six of the eleven patients recovered. Septicaemia was responsible for death in three, hepatic failure in one and methaemoglobinaemia in another. It is postulated that release of copper from haemolysed red cells during acute haemolytic episodes may initiate, or contribute to, the development of renal damage.. ...
We discuss a case of early RVT post-transplantation with delayed recovery after immediate open thrombectomy. RVT is generally a catastrophic complication of kidney transplantation, often managed with immediate allograft nephrectomy [1, 2, 17, 18]. Published reports describing cases managed with thrombectomy usually give little information regarding the intra-operative findings or the procedure performed, and often do not detail the duration of oligoanuria post-thrombectomy after which the allograft was deemed non-salvageable [1, 19, 20]. Of the three case reports describing successful thrombectomy that also describe the post-operative course, all three patients were independent from dialysis within 1 week [4, 21]. Our patient demonstrated remarkable renal reserve and regenerative capacity, eventually gaining independence from dialysis 2 weeks post-thrombectomy despite prolonged oliguria and the development of antibody-mediated rejection. We speculate that if a good outcome could be obtained with ...
Background: Pregnancy-related acute kidney injury (PRAKI) contributes to 3-7% of overall acute kidney injury (AKI) cases in Indian subcontinent. It reflects the absence of prenatal care and early detection of high-risk pregnancies, the delay in transfer of patients and the paucity of relevant human and material resources. It is certainly a treatable and curable complication, but one that imposes a heavy burden of maternal morbidity and mortality if its diagnosis and treatment are delayed. The best treatment remains prevention, a goal very difficult to attain in the developing countries.Materials and methods: AKI was diagnosed when there was a history of sudden oliguria (urinary output , 400 ml over 24 hrs or less than 20ml/hour) or anuria with a sudden increase in serum creatinine to more than 1.5mg/dl or an increase in serum creatinine of , 0.5mg/dl/day from baseline. All patients with obstetrical AKI, antepartum as well as postpartum, were included in this study. Results: Out of 40 patients ...
1)Hyperkalemia can lead to heart arrest, which is the most dangerous complication of Acute Renal Failure. Moreover, the patients also have hyponatremia, hyperphosphatemia and hypocalcemia. The main cause of hyperkalemia is oliguria or anuria. Moreover, serious infection, bleeding or hemolysis also can lead to hyperkalemia. In addition, hyponatremia, hypocalcemia and acidosis aggravate hyperkalemia to some extent. Hyperkalemia is the main death cause for the patients with Chronic Renal Failure. Therefore, the patients should pay more attention to it and adopt treatment in time ...
Multiorgan failure (MOF) due to intoxication, trauma or sepsis in the progressive late stages always include acute renal failure (ARF). The prognosis of these patients is poor despite adequate dialysis. This study included 27 consecutive patients (20 men and 7 women, age range 15-77 years) with a rapid progress of MOF including ARF, who were treated by plasma exchange as an attempt to reverse the progress of MOF. Twenty-three of the patients suffered from a septic shock. Oliguria or anuria was present in all, dialysis was performed in 16 of them, and mechanical respiratory aid in 17. Plasma exchange was performed 1-10 times and almost exclusively by centrifuge technique, using albumin and/or liquid stored plasma (in a few cases fresh frozen plasma) as colloidal replacement fluid. Twenty-two patients survived (81%) and 5 patients died. The reasons of death were cerebral haemorrhagia, brain abscess, myocardial sudden death, relapsing sepsis from multiple hepatic abscesses and a not drained psoas ...
Renal failure is also called renal inefficiency this is a medical condition which occurs when kidney fail to filter waste products and toxics out of the blood. Two forms of kidney injury are acute kidney failure and chronic kidney failure. These diseases are not recognized easily so Renal failure diagnosis is very necessary to cure this disease.. In acute kidney disease, there occurs rapidly progressive loss of renal functions. It is generally categorized by electrolyte imbalance, oliguria and fluid. There are many causes result in acute kidney failure. Whatever the cause is, it should identify as fast as possible and renal treatment should be started to stop the progress of the disease. Dialysis is also necessary. Chronic kidney disease develops slowly and it shows some symptoms that are not enough to conclude that a person is having chronic kidney disease. There occurs a condition when acute kidney injuries come in the top of chronic kidney disease that is called Acute-on-Chronic renal failure ...
To the editor: Reversible acute renal failure with oliguria and azotemia are known to occur as rare side effects of most nonsteroidal anti-inflammatory drugs (1-3). Occasional case descriptions of hematuria, papillary necrosis, and micturition disorders have also been recorded. Curt and associates (4) have reported a case of acute renal failure with nephrotic syndrome accompanied with features of fever, elevated sedimentation rate, and eosinophilia secondary to a hypersensitivity reaction to fenoprofen calcium. My observations on the following two cases are pertinent to their report.. A 72-year-old woman received fenoprofen calcium (Nalfon; Eli Lilly & Company, Indianapolis, Indiana), 600 mg three ...
TY - JOUR. T1 - Management and follow-up of arterial thrombosis in the neonatal period. AU - Payne, R. Mark. AU - Martin, Thomas C.. AU - Bower, Richard J.. AU - Canter, Charles E.. PY - 1989/5. Y1 - 1989/5. N2 - The management and follow-up of 12 patients with major aortic thrombus formation occurring in the neonatal period between 1982 and 1987 are reported. Umbilical arterial catheters were inserted in 8 of the 12 patients before thrombus formation. Two patients had congenital thrombl. Hypertension, oliguria, hematuria, and elevated blood creatinine concentration were found at the time of diagnosis of the thrombus; nine of the patients had a patent ductus arteriosus. Supportive care was instituted in seven patients who were hemodynamically stable. Five of the patients had congestive heart failure, shock, or both, and were treated with surgical thrombectomy. Thrombolytic therapy was not used in either group. The five surgically treated patients and six of seven medically treated patients ...
Within a fortnight in November 2008, 34 Nigerian children, aged 4 months to 3 years, died and more than 50 others were hospitalised with severe kidney damage after taking the drug "My Pikin" ("my child" in local pidgin), a teething mixture containing paracetamol.1 The outbreak was due to the use of diethylene glycol (DEG)2 as a solvent for the paracetamol. DEG was present because of inadvertent or deliberate substitution of propylene glycol, which is much less toxic than DEG and is widely used in the pharmaceutical industry.3 DEG is a colourless and odourless liquid, commonly used in industry, and can be found in commercial products such as resins, antifreeze, inks and glues.4 It is a well-known human toxicant that causes generalised multi-organ failure characterised by acute renal failure (oliguria or anuria) frequently accompanied by severe neurological dysfunction (cranial nerve palsies, acute … ...
In Chinese medical form, Chinese medicine is widely used in the treatment of various kidney diseases. In addition, many natural treatments have been developed based on Chinese medicine. And the therapy of purifying toxins from the blood takes on the best effects, helping patients reduce high levels of creatinine. From many years of clinical experience, if waste products and toxins are eliminated from the body, firstly, patients can often act better than those who do not eliminate waste products, since toxins in the blood not only cause additional damage to kidney and tissue cells , and also affect the effectiveness of drugs for the treatment of the kidneys. Because the toxins deposited in the blood not only cause additional damage to the kidney function, but also affect the effectiveness of treatment of kidney drugs. Therefore, this treatment mainly uses medicinal herbs that help patients to isolate products from waste and toxic substances. When using herbal medicines, various natural products ...