Our laboratory studies the host-pathogen relationship between viruses and cells to better understand cellular and molecular mechanisms underlying the complexities of viral disease. In particular, we study the human JC polyomavirus, which infects the majority of the population and causes the fatal demyelinating disease progressive multifocal leukoencephalopathy in the brain of immunosuppressed individuals. Our goal is to understand the basic biology of the virus and JCPyV pathogenesis in order to develop novel treatment options, which are currently very limited.. The Maginnis laboratory is committed to excellence in biomedical research and educational training. We aim to provide a collegial, supportive atmosphere that promotes a passion for scientific curiosity and research excellence in an environment where scientific research is fun and rewarding. We emphasize the importance of asking key scientific questions with creative, open minds to provide meaningful contributions in the fields of ...
JC virus (JCV) is a common human polyomavirus responsible for the fatal demyelinating disease progressive multifocal leukoencephalopathy (PML), in immunocompromised individuals. About 5% of AIDS patients develop this currently untreatable fatal disease. Typical and atypical antipsychotic drugs inhibit JCV infection of glial cells. Elphick et al. now find that the cellular receptor for JCV on glial cells is a serotonin receptor. The findings contribute to the understanding of the pathogenesis of PML in AIDS patients and suggest that therapy based on existing serotonin receptor inhibitors may be feasible.. G. F. Elphick, W. Querbes, J. A. Jordan, G. V. Gee, S. Eash, K. Manley, A. Dugan, M. Stanifer, A. Bhatnagar, W. K. Kroeze, B. L. Roth, W. J. Atwood, The human polyomavirus, JCV, uses serotonin receptors to infect cells. Science 306, 1380-1383 (2004). [Abstract] [Full Text]. ...
Progressive multifocal leukoencephalopathy (PML) is a fatal demyelinating disease caused by human neurotropic JC virus. JCV infects more than 80% of human popul...
One of the major limitations of highly active antiretroviral therapy is its inability to inhibit the replication of polyomavirus JC (JCV), the etiologic agent of progressive multifocal leukoencephalopathy (PML), an acquired immunodeficiency syndromeâ€"defining illness. We previously demonstrated the induction of interferon (IFN)â€"stimulated genes (ISGs) by JCV. In the present study, we characterize the specific viral events required to induce ISGs and the potential antiviral effects of type I IFN on JCV replication in human fetal glial cells in the presence and absence of type I IFNs. Productive JCV replication was essential for the induction of the antiviral host response. JCV replication at all steps was significantly inhibited in the presence of IFN, and neutralizing anti-IFN antibody rescued the inhibitory effect of IFN. These results support the use of IFN as an adjunct therapy for patients with PML. Because IFN cannot cross the blood-brain barrier to achieve its direct antiviral ...
JC virus can cause the serious neurological disease progressive multifocal leukoencephalopathy (PML) in patients who are immunocompromised. Presence of JC virus antibodies alone is not enough to diagnose PML, however, because the majority of adults who have been exposed to the virus dont develop symptoms
John Cunningham virus (JCV) constitutes a family of polyoma viruses, which plays important roles in the progressive multifocal leukoencephalopathy (PML) and tumorigenesis. However, no bibliometric investigation has been reported to guide the researchers and potential readers. Papers were collected from database Sci-expanded and Pubmed until May 22, 2008. The highly-productive authors, institutes and countries, highly-cited authors and journals were ranked. The highly-cited articles were subjected to co-citation and chronological analysis with highly-frequent MeSH words for co-occurrence analysis. Until now, 1785 articles about JCV were indexed in Sci-expanded and 1506 in Pubmed. The main document type was original article. USA, Japan and Italy were the largest three producers about JCV. Temple University published 128 papers and ranked the top, followed by University of Tokyo. Khalili K and Yogo Y became the core authors due to more than 20 documents produced. Journal of Neurovirology published more
The JC virus belongs to the polyoma virus group of the papovavirus family, which are double-stranded DNA viruses without an envelope. BK virus is also in the polyoma virus group. It appears that infection by both viruses occurs during childhood or adolescence, with about 50% of the population demonstrating antibody before adulthood, rising later to 80%-90%. The JC virus localizes to and remains latent in the kidney, from whence it occasionally may reactivate. If a patient becomes immunosuppressed, especially during AIDS, reactivated JC virus can infect lymphocytes, be carried to the brain, infect oligodendroglia glial cells, and produce a demyelinating disease called progressive multifocal leukoencephalopathy. This occurs in about 4% of patients with AIDS. Diagnosis is made through biopsy using immunologic stains containing antibody against polyomavirus. One report applied a homemade nucleic acid probe with PCR amplification to urine of JC virus patients and obtained an excellent detection ...
Treatment with natalizumab in patients with multiple sclerosis (MS) appears linked with JC virus (JCV) infection, which can lead to a rare and often fatal demyelinating disease of the central nervous system called progressive multifocal leukoencephalopathy (PML) that destroys the myelin that protects nerve cells.
Human JC polyomavirus (JCV) causes progressive multifocal leukoencephalopathy (PML), a rare but life-threatening demyelinating disease in patients under profound immunosuppression and those receiving therapeutic immunomodulation for autoimmune and inflammatory disorders. Using persistent mouse polyomavirus (MPyV) infection in mice, we are developing a model of PyV-induced CNS disease to elucidate the role of immunosuppression in PML pathogenesis. Histology revealed corpus callosum demyelination in 100% of persistently infected mice. Following intracerebral injection with MPyV, immunocompetent C57BL/6 mice showed robust infiltration by MPyV-specific CD8 T cells with minimal contraction. During persistent CNS infection, a significant population of CD8 T cells exhibited a tissue-resident memory (Trm) phenotype (CD103+CD69+CD62Llo), as reported for acute VSV and latent HSV-1 infections, and incorporated BrdU in situ, suggesting capacity for self-renewal. Furthermore, these Trm cells develop and ...
On my website I have invited medical practitioners and other health professionals to debate the information that I am providing on Australias vaccination policies. John Cunningham decided to take up this offer in February/March 2014. He is a leader of the Stop the Australian Vaccination Network (SAVN) lobby group and a medical practitioner. When responding to my request he chose to send his opinions in unsolicited emails to members of the public, including UOW academics involved with my research and journalists. This occurred whilst I was a student at the University of Wollongong (UOW).. Here are the corrections to the personal opinions and misinformation provided by John Cunningham in unsolicited emails to members of the public and UOW academics in 2014.. Further, in July 2014 John Cunningham, made a complaint to the University of Wollongong about my research project completed in 2006. This research project was a critique of the Australian governments whooping cough policy and the research ...
Igor Koralnik, MD is an American physician, neurologist and scientist. He is one of the first physicians to study the neurologic complications caused by the human immunodeficiency virus (HIV) and is a leading researcher in the investigation of the polyomavirus JC (JC virus), which causes progressive multifocal leukoencephalopathy (PML), a disease of the central nervous system that occurs in immunosuppressed individuals. Koralnik was born in Geneva, Switzerland on July 20, 1962. He moved to the United States in 1990 and became a naturalized U.S. citizen. He received his medical degree at the University of Geneva Medical School, Switzerland, in 1987. While a medical student, he became interested in a new disease - acquired immune deficiency syndrome (AIDS), whose origin in the HIV virus was discovered in 1983. Koralniks doctoral dissertation focused on the early neurological complications of AIDS. He used magnetic resonance imaging, electroencephalography and other electrophysiologic diagnostic ...
the jc virus, or john cunningham virus, is a germ so common that the majority of adults have been exposed to it. the virus was first discovered in 1971, when a doctor found it in the brain of a man wi
For a comprehensive list of publications, see the PubMed link above. Below is a selection of publications:. Cabral HJ, Tobias C, Rajabiun S, Sohler NL, Cunningham CO, Wong MD, Cunningham WE. Outreach program contacts: Do they increase the likelihood of engagement and retention in HIV primary care for hard-to-reach patients? AIDS Patient Care STDs. 2007 Jun;21(S1):S59-S56.. Cunningham CO, Kunins HV, Roose RJ,Elam RT, Sohler NL. Barriers to Obtaining Waivers to Prescribe Buprenorphine for Opioid Addiction Treatment Among HIV Physicians. J Gen Intern Med. 2007 Sep;22(9):1325-9. Epub 2007 Jul 10. PMC2219773. Cunningham CO, Sanchez JP, Heller D, Sohler NL. Assessment of a medical outreach program to improve access to HIV care among marginalized individuals. Am J Public Health. 2007 Oct;97(10):1758-61. Epub 2007 Aug 29. PMC1994196. Cunningham CO, Giovanniello A, Sacajiu G, Whitley S, Mund P, Beil R, Sohler NL. Buprenorphine treatment in an urban community health center: What to expect. Fam Med. 2008 ...
The JC virus is a virus which infects the brain of advanced cancer patients and causes PML-- progressive multifocal leukoencephalopathy. PML can lead to neurologic deficits as indicated in your...
Background and aims: Anti-α4 integrin therapy with natalizumab is efficacious in refractory Crohns disease and in multiple sclerosis, but carries an estimated 1/1000 risk of progressive multifocal leukoencephalopathy (PML) caused by reactivation of latent JC virus infection. Athough anti-α4 integrin therapies are likely to be introduced in the clinic, screening for the risk of PML has not been developed.. Methods: We prospectively collected urine, serum, plasma and buffy coats from 125 patients with Crohns disease, 100 control subjects with gastrointestinal (GI) disease, and 106 healthy volunteers. Four to eight weeks after this first sample collection, we additionally collected a set of urine, serum, plasma and buffy coat samples from the 125 patients with Crohns disease, and a next set of samples was collected 12-16 weeks after the first collection. JC viral loads were determined with quantitative real-time polymerase chain reaction (PCR), and JC virus seroprevalence with a specific ...
The initial site of infection may be the tonsils,[4] or possibly the gastrointestinal tract.[5] The virus then remains latent in the gastrointestinal tract[6] and can also infect the tubular epithelial cells in the kidneys,[7] where it continues to reproduce, shedding virus particles in the urine. JCV can cross the blood-brain barrier into the central nervous system, where it infects oligodendrocytes and astrocytes, possibly through the 5-HT2A serotonin receptor.[8] JC viral DNA can be detected in both non-PML affected and PML-affected (see below) brain tissue.[9] JCV found in the central nervous system of PML patients almost invariably have differences in promoter sequence to the JCV found in healthy individuals. It is thought that these differences in promoter sequence contribute to the fitness of the virus in the CNS and thus to the development of PML.[3] Certain transcription factors present in the early promoter sequences of the JC virus can induce trophism and viral proliferation that ...
Rapid restoration of virus-specific T immunity via adoptive transfer of ex vivo generated T cells has been proven as a powerful therapy for patients with advanced cancers and refractory viral infections such as cytomegalovirus (CMV) and Epstein-Barr virus (EBV). BK virus (BKV), John Cunningham virus (JCV), and Merkel cell carcinoma virus (MCV) are the members of the rapidly growing human polyomavirus (hPyV) family that commonly infects most healthy humans. These viruses have a clearly established potential for causing severe end-organ damage or malignant transformation, especially in individuals with weakened immunity who are unable to mount or regain endogenous T-cell responses as a result of underlying leukemia or iatrogenic immunosuppression in autoimmunity, bone marrow and solid organ transplant settings ...
Of 41 HIV+ patients, 38 were male (93%) and indications for BM biopsies were lymphoma (20), pancytopenia (8), anemia (6), MGUS (2), polycythemia (2), leucopenia (1), splenomegaly (1), and thrombocytopenia (1). Mean age was 47 (range 32-71), mean CD4 count 211 (2-928), 27% had undetectable plasma HIV RNA, and 75% were on HAART. The 47 HIV- controls, matched for indications for BM biopsy, included 32 males (65%), and mean age was 56 (range 27-93). Quantitative PCR detected JCV DNA in BM samples of 19/41 (46%) HIV+ vs 3/47 (6%) of HIV- (p , 0.001). Preliminary immunohistochemistry (IHC) experiments suggest that JCV T antigen is detectable in a fraction of JCV DNA positive bone marrows samples, while JCV VP1 protein was not. Furthermore, JCV was detected by double IHC in some of the plasma cells, myeloid, and lymphoid cells. We then tested fresh BM aspirates, blood and urine samples from 30 HIV- and 6 HIV+ patients. JCV DNA was detectable in 10/36 (28%) fresh BM aspirates, 7/26 (27%) peripheral ...
Online etext Cunninghams manual of practical anatomy (Volume 1) by D. J. (Daniel John) Cunningham (page 7 of 44) : the collection of free ebooks
Once Dr. Cunningham discovered Ophthalmology, he says, there was no second-guessing. He knew he had found his passion, and was determined to use his medical knowledge, curiosity about advanced technologies, and genuine interest in others to help people of all ages achieve the best possible vision. In 1991, Dr. Cunningham joined the Knoxville Ophthalmology practice started by Dr. J. Ed Campbell in 1952, and has been an integral part of a number of firsts achieved by the practice ever since, including: first in the Southeast to perform the Crystalens® procedure, a cataract replacement lens that is also designed to correct vision at all distances and East Tennessees first onsite Custom LASIK surgical facility, the Campbell Cunningham Laser Center.. While hes accomplished a lifetime of goals in less than 25 years in Ophthalmology, Dr. Cunningham is quick to point out that in his precision, technology-based specialty, there is always something new to learn and accomplish. Some of his best teachers ...
Human polyomaviruses (JC virus, BK virus and simian virus 40) are causative agents of some human diseases and, interestingly, are involved in processes of cell transformation and oncogenesis. These viruses need the cell cycle machinery of the host cell to complete their replication; so they evolved mechanisms that can interfere with the growth control of infected cells and force them into DNA replication. The retinoblastoma family of proteins (pRb), which includes pRb/p105, p107 and pRb2/p130, acts as one of the most important regulators of the G1/S transition of the cell cycle. Rb proteins represent an important target for viral oncoproteins. Early viral T antigens can bind all members of the pRb family, promoting the activation of the E2F family of transcription factors, thus inducing the expression of genes required for the entry to the S phase. The interaction between early viral antigens and cell cycle regulators represents an important mechanism through which viruses deregulate cell cycle ...
The JC virus, or John Cunningham virus, is a member of the polyomavirus family. When youre exposed to it as a kid or young adult, you might not even notice it, or you might notice cold-like symptoms which are self-limiting. Not a huge deal at all. You get infected, you make antibodies, life goes on. As long as youre immunocompetent. But thats not the case in patients with impaired cellular immunity, as in cases of Hodgkins lymphoma (HL).. Although the gold standard for diagnosis is brain biopsy, you dont need tissue to make the diagnosis of PML. In an immunocompromised patient with progressive neurologic changes, youll always need the MRI. This can exclude opportunistic processes like toxoplasmosis, cryptococcal meningitis, or CNS lymphoma, but will also be important to confirm the presence of white matter disease, which may or may not have enhancement 10-20% of the time, and may or may not have mass effect. Our patient had faint enhancement, as you can see from the images on the blog. Once ...
Cunningham earned an undergraduate degree at Mississippi State College for Women and a Ph.D. in both microbiology and immunology from the University of Tennessee Center for Health Sciences.. Today, she is a George Lynn Cross Research Professor and Presbyterian Health Foundation Presidential Professor in the Department of Microbiology and Immunology at the University of Oklahoma College of Medicine, where she has taught and conducted research into infection and autoimmune diseases for 33 years.. "My training was in Group A streptococcal disease, but I also love immunology, which is our bodys response to infections," she said.. After researching rheumatic fever, myocarditis and other heart-related diseases for 15 years, Cunningham received a call from the National Institutes of Mental Health. They wanted her to investigate a puzzling condition affecting children known as pediatric autoimmune neuropsychiatric disorder associated with strep, commonly known as PANDAS.. Cunningham became a ...
Cunningham, rundles S.; Feller, W F.; Cunningham, rundles C.; Dupont, B; Wanebo, H; Oreilly, R; and Good, R A., "Lymphocyte transformation in vitro to riii mouse milk antigen among woman with breast disease." (1976). Subject Strain Bibliography 1976. 2295 ...
Prosecutors allege Cunningham violated a law that forbids disclosing photos or videos without the consent of someone "whose intimate parts are exposed" or who is engaged in a sexual act. They say the woman and Cunningham lived together.. ...
Dr. Matthew Cunningham, MD of New York, NY patient reviews, appointments, phone number and quality report. Compare Dr. Cunningham to other nearby Orthopedic Surgeons in New York.
Scott stopped by Ward Cunningham (you know, the guy who invented the Wiki. Yes, that Ward Cunningham) to hang out, and discovered Wards treasure trove of electronics, software, soldering guns and we
Issuu is a digital publishing platform that makes it simple to publish magazines, catalogs, newspapers, books, and more online. Easily share your publications and get them in front of Issuus millions of monthly readers. Title: Paul James Cunningham // Untitled 2.0 // Geometry of Fear, Author: Untitled 2.0, Name: Paul James Cunningham // Untitled 2.0 // Geometry of Fear, Length: 106 pages, Page: 1, Published: 2017-12-23
Obituary for Joan Cunningham Heidelberg | JOAN CUNNINGHAM HEIDELBERG, age 87, of Troy, Ohio passed away on Friday, August 14, 2020 at Hospice of Miami County Inpatient Unit in Troy surrounded by love. She was born on October 27,...
The principal CNS targets for JCV infection are oligodendrocytes (the brain cells that produce myelin). A stable human oligodendrocyte cell line for studies of JCV dependent PML was, however, not previously available. Therefore, we were delighted to report in Peterson et al, 2017 that a human stem cell line termed G144 (Pollard et al, 2009) undergoes differentiation by removal of growth factors (Fig. 1A), express oligodendrocyte specific surface markers (Fig. 1B), has a rapid doubling time (3-5 days) and is stable during passage. Furthermore, we reported that G144 cells support robust levels of JCV DNA replication and infection by JC virus. Thus, we have established the first oligodendrocyte based model system for studies of JCV dependent PML.. ...
Progressive brain disorder that slowly destroys a persons ability to learn, reason, make judgments, communicate and perform daily activities. As this disease progressives the individuals may experience memory loss, this usually occurs more in the short-term memory, changes in personality and behavior, such as anxiety, suspiciousness or agitation, as well as delusions or hallucinations. Brain cells are gradually destroyed and leads to progressive decline in mental functions, No cure is available at this time but their are treatments that can slow the progress of the disease.
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Different studies suggested an oncogenic potential of the polyomaviruses JC virus, BK virus and simian virus 40, particularly in brain tumors and neuroblastoma, which belong to the most frequent malignancies in children. However, currently available data are controversial, possibly due to the different regional prevalence of the viruses and the detection techniques used.
While it would be extremely concerning if the patient in question developed PML while her lymphocyte counts remained at acceptable levels, the facts as now known indicate that this simply was not the case. Given the immunosuppressive properties of Tecfidera, it would appear to be vitally important that patients be regularly tested to check their white blood cell counts. It would seem that the FDA suggestion of once yearly blood tests is insufficient, and I know many neurologists are checking their patients much more frequently, more on the order of at least once every three months. Monitoring for JC virus might make sense if PML were to be seen in Tecfidera patients not experiencing severe lymphopenia, but at this point the available evidence doesnt seem to merit the taking of this extra step. Then again, any extra bit of information is valuable, and Id imagine that some neurologists might Institute testing for JC virus in their Tecfidera patients on their own. Tecfidera is proving to be quite ...
Specimen Type: Formalin-fixed, paraffin-embedded tissue block. Supplies: Pathology Packaging Kit (T554). Specimen Volume: Entire block. Specimen Type: Slides. Slides: 4 Unstained glass, positively charged slides with 5 (+ or - 1)-microns formalin-fixed, paraffin-embedded tissue. ...
Verma S, Ziegler K, Ananthula P, Co J K G, Frisque R J, Yanagihara R, Nerurkar V R (2006); Virology, 345:457-467. doi: 10.1016/j.virol.2005.10.012. ...
Cunningham, Patricia https://edrn.nci.nih.gov/sites/5-fred-hutchinson-cancer-research-center/cunningham-patricia https://edrn.nci.nih.gov/logo.png ...
Bard Cunningham Clamp is used for controlling enuresis, stress incontinence, or dribbling of urine in males of all ages. Reusable. FSA Approved. Shop Now!
IMOGEN CUNNINGHAM (American, 1883-1976) Nude, 1932 Gelatin silver, circa 1950 6-3/4 x 8-7/8 inches (17.1 x 22.5 - Available at 2011 May New York Signature...
Like other vehicles in its class, it accelerated faster than the automobiles most people drive and shifted through its gears while reaching rpms of 30,000 to 40,000.Simply put, it and the other radio-controlled race cars of the Queens United R/C Club may only be a foot long, but childrens toys they are not."Anything you can get for your car you can get for one of these cars," said club member Frank Freni, 36, of Queens Village.Every Sunday from April to October, and some Saturdays, too, radio-controlled-car enthusiasts from around the borough gather in a Cunningham parking lot under the auspices of Queens United to run their cars, work on their equipment and occasionally race against each other.The meetings, now in their second season, are comprised of a dozen club members who have each paid a $50 yearly fee, plus additional hangers-on and passers-by. Queens United was founded last year by Mark Salonger, 47, a former computer network engineer from Forest Hills, who became disabled after a ...
Macular degeneration is a disease that causes reduction in central vision. The patient still has peripheral or side vision, but central vision-where we see the clearest, sharpest images-slowly diminishes.. The macula is a small nerve layer on the retina. Its located in the back of the eye, and is only about the size of a pinhead. But this nerve layer is essential to our seeing fine detail. As macular degeneration progresses, it slowly causes the ability to see details to disappear. Images may appear distorted, or parts of them may be missing. The disease may slowly continue to progress, robbing the patient of critical visual ability.. The cause of macular degeneration is uncertain though heredity, age and general health may be factors. Exposure to ultraviolet light, smoking or other environmental factors may also contribute. Currently, there is no cure. But, there are new treatments for the disease that are showing promise. Drs. Campbell, Cunningham, Taylor & Haun offer comprehensive care for ...
VISION Director Jacob Cunningham said the deadline was extended to 11:59 p.m. Friday, Oct. 21, because the program doesnt have enough people signed up.. "We didnt get the number we wanted to fill our trips," Cunningham said. "This year we are offering more trips than in years past, so we have more spots available.". Cunningham attributes a decline in this years applicants to the current economy.. "There is less money to do things that dont count towards your degree," Cunningham said.. Freshman Kate Kollmann said this is whats holding her back.. "If I was going on a J-term trip and paying that money, I would want to make sure I got a class (credit) for it," Kollmann said. "That is the only drawback for me.". Junior Billy Kor agrees.. "The only thing that really hinders me is the cost," Kor said.. However, senior Craig Spooner cites other reasons, "I have thought about going on trips before a bunch of times. Its not like it costs a bunch of money… it just didnt work out for me at the ...
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Adeline Cunningham was born in 1852 and held in bondage by Washington Greenlee Foley on his vast Texas corn and cotton plantation. She described to the interviewer the horrendous, inhumane conditions of life on Foleys large farm, where slaves were treated as if they were animals and brutally punished for trying to escape. Prints and Photographs Division; Manuscript Division, Library of Congress deys afraid iffen de slaves learns to read dey learns how to run away. One of de slaves runs away and dey ketches him and puts his eyes out.
Dr. Paul Y. Cunningham III is a specialist in Preventive Cardiology, Vascular Medicine, Interventional Cardiology, Cardiology who can be reached at 936.270.3933 and whose practice locations include: The Woodlands, Conroe
Eric Shane Cunningham DO is a Internal Medicine who practices in Montgomery, AL. Get a full report about this doctors background by clicking here.
There were a number of issues raised at the Rewa Provincial Council meeting, but the sewage spill at the Cunningham Creek topped the agenda. Discussions heard that the spill had...
10th Floor BJCIH AB Conference Room, Fridays at 10:30 a.m.-12:00 p.m.. June 6th First session: 1) Dr. Indira Mysorekar introduction and presentation and 2) Emily Ma (Mysorekar Lab) (JC). June 13th Second session: 1) Dr. Jeff Miner presentation and 2) Jake Foote (Miner Lab) (JC). June 20th Third session: 1) Michelle Recto (Jain Lab) (JC) and 2) Joy Twentyman (Mysorekar Lab) (JC). June 27th Fourth session: 1) Dr. Vijay Samineni presentation and 2) Maya Menon (Jain Lab) (JC). July 11th Fifth session: 1) Dr. Sanjay Jain presentation and 2) Chiragh Kapadia (Jain Lab) (JC). July 18th Sixth session: 1) Dr. Judy Golden presentation and 2) John Webb (Golden Lab) (JC). July 25th Seventh session: 1) Grace Deitzler (Lewis Lab) (JC) and 2) Connelly Miller (Henderson Lab) (JC). August 1st Eighth session: Student summer research presentations. August 8th Ninth session: Student summer research presentations continued. August 15th Tenth session: 1) Colin Macones (Mysorekar Lab) (JC) and 2) Akhil Kumar (Mysorekar ...
Kondo Y, Windrem MS, Zou L, Chandler-Militello D, Schanz SJ, Auvergne RM, Betstadt SJ, Harrington AR, Johnson M, Kazarov A, Gorelik L, Goldman SA. "Human glial chimeric mice reveal astrocytic dependence of JC virus infection." The Journal of clinical investigation.. 2014 Dec 0; 124(12):5323-36. Epub 2014 Nov 17. 2012 ...
Cruise summary report for RRS James Cook JC039 (AMT19, JC040) - produced from a first level inventory of measurements and samples collected at sea that provides information on who has collected what, where and when. Assembled initially from cruise plans and then updated from other sources including the Intergovernmental Oceanographic Commission (IOC) Cruise Summary Report (ROSCOP) forms submitted to BODC for each UK research cruise. Provides a useful resource of cruise activities to scientists, programme managers and data managers.