The present study shows, for the first time, the time-dependent and LDL-R phenotype-dependent effect of high-dose atorvastatin on HDL cholesterol levels in patients with HFH. Our major findings are that (1) an early and transitory reduction of HDL cholesterol levels occurs during the first weeks of atorvastatin treatment in HFH patients; (2) the degree of the transitory reduction is greater in receptor-negative patients than in receptor-defective patients; and (3) after long-term treatment, HDL cholesterol concentration remains higher in receptor-defective than in receptor-negative patients as a function of residual LDL-R activity in receptor-defective HFH patients.. Consistent with previous reports,5,6 we did not find a significant variation in mean HDL cholesterol levels after long-term treatment of HFH patients with high-dose atorvastatin. However, we confirmed in those patients a short-term reduction in HDL cholesterol levels that was reported in heterozygous FH patients,3 with the nadir ...
TY - JOUR. T1 - High-dose atorvastatin enhances impaired cerebral vasomotor reactivity. AU - Forteza, Alejandro. AU - Romano, Jose G.. AU - Campo-Bustillo, Iszet. AU - Campo, Nelly. AU - Haussen, Diogo C.. AU - Gutierrez, Jose. AU - Koch, Sebastian. PY - 2012/8/1. Y1 - 2012/8/1. N2 - The influence of statin therapy on cerebral vasomotor function has not been fully characterized. We report the effects of high-dose atorvastatin therapy on cerebral vasomotor reactivity (VMR) in patients with controlled hypertension and dyslipidemia. We prospectively enrolled 36 patients with controlled hypertension and a low-density lipoprotein (LDL) cholesterol concentration ,100 mg/dL. Atorvastatin 80 mg was given daily for 6 months and then discontinued. VMR was assessed by hypercapnic and hypocapnic transcranial Doppler challenge in both the right and left middle cerebral artery (MCA) at baseline, and after 3 and 6 months of therapy. Forty-five days after statin cessation, a repeat VMR was performed. VMR ...
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ACTG A5275 was a prospective, double-blind, randomized, placebo-controlled cross-over design pilot study evaluating the effect of atorvastatin on biomarkers of inflammation, coagulopathy, angiogenesis, and T-lymphocyte activation in HIV-1 infected individuals with suppressed HIV-1 RNA on stable protease inhibitor based antiretroviral therapy with fasting LDL cholesterol , 130 mg/dL.. Atorvastatin is a drug approved by the Food and Drug Administration (FDA) for treating high cholesterol. Atorvastatin has also been able to lower the level of inflammation blood tests in certain other diseases but has not been studied for this purpose in people who have HIV. The main goal of this experimental study is to see how taking atorvastatin affects inflammation blood tests in people infected with HIV who do not need to take medicine for high cholesterol. In addition to observing the effects of atorvastatin on the level of inflammation measured in the blood, this study evaluated if atorvastatin is safe for ...
ACTG A5275 was a prospective, double-blind, randomized, placebo-controlled cross-over design pilot study evaluating the effect of atorvastatin on biomarkers of inflammation, coagulopathy, angiogenesis, and T-lymphocyte activation in HIV-1 infected individuals with suppressed HIV-1 RNA on stable protease inhibitor based antiretroviral therapy with fasting LDL cholesterol , 130 mg/dL.. Atorvastatin is a drug approved by the Food and Drug Administration (FDA) for treating high cholesterol. Atorvastatin has also been able to lower the level of inflammation blood tests in certain other diseases but has not been studied for this purpose in people who have HIV. The main goal of this experimental study is to see how taking atorvastatin affects inflammation blood tests in people infected with HIV who do not need to take medicine for high cholesterol. In addition to observing the effects of atorvastatin on the level of inflammation measured in the blood, this study evaluated if atorvastatin is safe for ...
The study by Cannon and colleagues shows that more intensive lowering of low-density lipoprotein (LDL) cholesterol with statin therapy in patients with a recent ACS reduced subsequent risk for major adverse CV events. This trial also supports the hypothesis that more aggressive LDL reduction to a target level of 60 mg/dL (1.5 mmol/L) yields greater benefit than the current guideline recommendation (LDL level , 100 mg/dL [2.6 mmol/L]). In the ACS population, the benefit of very aggressive LDL reduction compared with moderate reduction is evident as early as 1 month after initiating therapy. This reduction would take 2 years in patients with stable coronary artery disease. In the MIRACL study, high-dose atorvastatin therapy initiated within days of an ACS diagnosis reduced recurrent ischemic events within 16 weeks (1). Early, aggressive statin therapy in patients with ACS has a powerful role in "passivating" the unstable and vulnerable plaque(s). The subgroup analysis in the study by Cannon and ...
Results Baseline demographic characteristics and nephropathy risk factors were similar between groups. 4.8% of patients in the high-dose group developed CIN versus 7.6% of those in the low-dose group (p,0.05). In the high-dose group, postprocedure serum creatinine was significantly lower (1.08±0.42 vs 1.14±0.38 mg/dl, p,0.05), creatinine clearance was decreased (78.9±22.2 vs 72.0±36.6 ml/min, p,0.05), multivariable analysis showed that high-dose atorvastatin pretreatment was independently associated with a decreased risk of CIN (OR 0.31, 95% CI 0.22 to 0.86, p,0.05).. ...
Objective: Lowering LDL-cholesterol (LDL-C) is associated with improved outcomes in patients at high risk for cardiovascular disease. LDL-C goals recommended by NCEP ATP III are dependent upon pre-existing cardiovascular risk factors. Statins are the first recommended pharmacotherapy. Since the availability of generic atorvastatin will likely increase its use, this analysis examined LDL-C goal attainment in high-risk US patients treated with atorvastatin monotherapy.. Methods: In this retrospective study using the GE Healthcare Centricity database, patients who received a prescription for atorvastatin monotherapy (the index Rx) between 01/01/08 and 12/31/10 were identified. They were selected for the analysis if they also had an ICD-9 diagnosis or CPT procedure code indicated for coronary heart disease or atherosclerotic vascular disease, ≥ 1 LDL-C measurement between 3 mo and 1 yr post index Rx, and medical records for 1 yr prior to and following index Rx. Endpoints included the proportion of ...
OBJECTIVE: This study aims to establish the benefits of lowering cholesterol in diabetic patients with well-controlled hypertension and average/below-average cholesterol concentrations, but without established coronary disease. RESEARCH DESIGN AND METHODS: In the lipid-lowering arm of the Anglo-Scandinavian Cardiac Outcomes Trial (ASCOT-LLA), 10,305 hypertensive patients with no history of coronary heart disease (CHD) but at least three cardiovascular risk factors were randomly assigned to receive 10 mg atorvastatin or placebo. Effects on total cardiovascular outcomes in 2,532 patients who had type 2 diabetes at randomization were compared. RESULTS: During a median follow-up of 3.3 years, concentrations of total and LDL cholesterol among diabetic participants included in ASCOT-LLA were approximately 1 mmol/l lower in those allocated atorvastatin compared with placebo. There were 116 (9.2%) major cardiovascular events or procedures in the atorvastatin group and 151 (11.9%) events in the placebo group
The use of short-term high-dose atorvastatin pretreatment is safe and significantly improves the final TIMI flow grade as well as reduces the 30-day MACEs in ACS patients post-PCI. This finding encourages the use of short-term high-dose atorvastatin pretreatment as an alternative for ACS patients undergoing PCI, but more high-quality randomized clinical trials are still needed to confirm the long-term efficacy and safety. ...
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The results of this analysis indicated that high-dose, compared with lower-dose, statin increased the risk of NOD among patients with 2 to 4 diabetes risk factors. No increased risk of NOD was seen with high-dose statin treatment in patients with 0 to 1 risk factors for diabetes. Slightly more than half of TNT patients and nearly two-thirds of IDEAL patients were in the low-risk group for diabetes. Compared with low-dose statin, atorvastatin 80 mg reduced the number of CV events both in patients at low and high risk for diabetes.. These results should reassure physicians treating patients at low risk for diabetes. Such patients do not appear to incur an increased risk of diabetes with high-dose atorvastatin and derive benefit in terms of CV event reduction. Among the 6,231 patients in the TNT and IDEAL trials at high risk for NOD, treatment with atorvastatin 80 mg compared with a lower statin dose was associated with 80 more cases of NOD and the prevention of 94 major CV events in 58 ...
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Other adverse reactions reported in placebo-controlled studies include:. Body as a whole: malaise, pyrexia; Digestive system: abdominal discomfort, eructation, flatulence, hepatitis, cholestasis; Musculoskeletal system: musculoskeletal pain, muscle fatigue, neck pain, joint swelling; Metabolic and nutritional system: transaminases increase, liver function test abnormal, blood alkaline phosphatase increase, creatine phosphokinase increase, hyperglycemia; Nervous system: nightmare; Respiratory system: epistaxis; Skin and appendages: urticaria; Special senses: vision blurred, tinnitus; Urogenital system: white blood cells urine positive. Anglo-Scandinavian Cardiac Outcomes Trial (ASCOT) In ASCOT [see Clinical Studies (14.1)] involving 10,305 participants (age range 40 to 80 years, 19% women; 94.6% Caucasians, 2.6% Africans, 1.5% South Asians, 1.3% mixed/other) treated with atorvastatin calcium 10 mg daily (n = 5,168) or placebo (n = 5,137), the safety and tolerability profile of the group treated ...
This study shows a substantial benefit with atorvastatin therapy in patients with systolic HF due to a nonischemic etiology. The LV systolic function improved significantly in the cohort of patients treated with atorvastatin, compared with a decline in systolic function in patients treated with placebo over the 12-month study period. In addition, there were reductions in both LV end-diastolic and end-systolic dimensions in the atorvastatin group when compared with placebo. These findings suggest that atorvastatin might retard the progression of adverse myocardial remodeling in patients with nonischemic HF. Finally, atorvastatin therapy was associated with reductions in levels of hsCRP, TNF-α RII, and IL-6 as well as an increase in E-SOD activity, suggesting an association between changes in pro-inflammatory and pro-oxidative markers and LV systolic function.. The patients in our study were largely either NYHA functional class II or III in symptoms. Baseline medical therapy was good, with 88% of ...
TY - JOUR. T1 - Enhanced cardioprotection against ischemia-reperfusion injury with combining sildenafil with low-dose atorvastatin. AU - Rosanio, Salvatore. AU - Ye, Yumei. AU - Atar, Shaul. AU - Rahman, Atiar M.. AU - Freeberg, Sheldon Y.. AU - Huang, Ming He. AU - Uretsky, Barry F.. AU - Birnbaum, Yochai. PY - 2006/2. Y1 - 2006/2. N2 - Purpose: Both ATV and SL reduce myocardial infarct size (IS) by enhancing expression and activity of NOS isoforms. We investigated whether atorvastatin (ATV) and sildenafil (SL) have synergistic effects on myocardial infarct size (IS) reduction and enhancing nitric oxide synthase (NOS) expression. Method: Rats were randomized to nine groups: ATV-1 (1 mg/kg/d); ATV-10 (10 mg/kg/d); SL-0.7 (0.7 mg/kg); SL-1 (1 mg/kg); ATV-1 + SL-0.7; water alone (controls); 1400W (iNOS inhibitor; 1 mg/kg); ATV-10 + 1400W; and ATV-1 + SL-0.7 + 1400W. ATV was administered orally for 3 days. SL was administered intraperitoneally 18 h before surgery and 1400W intravenously 15 min ...
This retrospective study investigated whether patients at a high risk of cardiovascular disease receiving atorvastatin monotherapy achieved guidelines-specific
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Background: High dose atrovastatin is known to be associated with new onset diabetes mellitus (DM) in patients (pts). However, there have been limited data whether low dose atrovastatin can be associated with increased incidence of new-onset DM. We investigated the impact of low dose (10mg or 20mg) atorvastatin on the development of new-onset DM based on five-year clinical outcomes in Asian patients.. Methods: We investigated a total of 3,566 consecutive patients (pts) who did not have DM from January 2004 to September 2009. To adjust potential confounders, a propensity score matched (PSM) analysis was performed using the logistic regression model. The primary end-point was the cumulative incidence of new-onset DM which was defined as having a fasting blood glucose ≥126 or HbA1c ≥6.5%. and assessed to determine the impact of low dose atrovastatin (10mg or 20mg).. Results: Mean follow-up duration was 976±278 days in all-pt group, and 993±240 days in PSM group. After PSM (C-statistics: ...
Other adverse reactions reported in placebo-controlled studies include: Body as a whole: malaise, pyrexia; Digestive system: abdominal discomfort, eructation, flatulence, hepatitis, cholestasis; Musculoskeletal system: musculoskeletal pain, muscle fatigue, neck pain, joint swelling; Metabolic and nutritional system: transaminases increase, liver function test abnormal, blood alkaline phosphatase increase, creatine phosphokinase increase, hyperglycemia; Nervous system: nightmare; Respiratory system: epistaxis; Skin and appendages: urticaria; Special senses: vision blurred, tinnitus; Urogenital system: white blood cells urine positive. Anglo-Scandinavian Cardiac Outcomes Trial (ASCOT) In ASCOT [see Clinical Studies (14.1)] involving 10,305 participants (age range 40 to 80 years, 19% women; 94.6% Caucasians, 2.6% Africans, 1.5% South Asians, 1.3% mixed/other) treated with atorvastatin calcium 10 mg daily (n=5,168) or placebo (n=5,137), the safety and tolerability profile of the group treated with ...
Background: Although the bio kinetics, metabolism and chemical toxicity of Atorvastatin are well known, until recently little attention was paid to the potential neurotoxic effect of Atorvastatin (Atv). Regarding the concrete evidences indicating Atv may reduce Coenzyme Q10 (CoQ10) levels through blockage of metalonate cycle, the present work aims to determine if Atorvastatin may provide toxic effects on brain mitochondria and whether its supplementation with two lipidicantioxidants, CoQ10 and Vit E may improve such outcomes. Methods: to evaluate mitochondrial toxicity, male NMRI mice were first treated with Atorvastatin(bo; 20 and 60 mg/kg) every other day with or without supplementation with CoQ10 (200 mg/kg) or Vit E (40 u/kg). After a period of 4 weeks, the animals were euthanized and brain cortices were harvested ad subjected to mitochondria isolation procedure. ROS release, mitochondrial membrane potential (MMP) and cytochrome c release were performed to precisely address the probable
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Background: Contrast-induced nephropathy (CIN) is a serious adverse event that certain patients experience as a result of a radiologic study performed with intravascular contrast media. The most at risk population includes patients with preexisting renal impairment. Due to the lack of successful treatment for contrast-induced nephropathy and its potentially critical results, preventative strategies are imperative. Though many approaches have been implemented, no universal standard protocol currently exists for the prevention of CIN in at-risk patients. Recent research has demonstrated controversial results of the use of short-term statin therapy for prevention of CIN. The aim of this systematic review is to evaluate the efficacy of atorvastatin in prevention of CIN in patients with renal impairment undergoing coronary angiography or percutaneous coronary intervention. Methods: An exhaustive search of available medical literature from MEDLINE-Ovid, MEDLINE-PubMed, and Google Scholar was performed using
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Objective The aim of the study was to evaluate a possible effect of atorvastatin on renal interleukins (ILs) and prostaglandin E2 (PGE2) in type 1 diabetic rats.
SABOGAL, Angélica María; ARANGO, César Augusto; CARDONA, Gloria Patricia e CESPEDES, Ángel Enrique. Atorvastatin protects GABAergic and dopaminergic neurons in the nigrostriatal system in an experimental rat model of transient focal cerebral ischemia. Biomédica [online]. 2014, vol.34, n.2, pp.207-217. ISSN 0120-4157. http://dx.doi.org/10.7705/biomedica.v34i2.1851.. Introduction: Cerebral ischemia is the third leading cause of death and the primary cause of permanent disability worldwide. Atorvastatin is a promising drug with neuroprotective effects that may be useful for the treatment of stroke. However, the effects of atorvastatin on specific neuronal populations within the nigrostriatal system following cerebral ischemia are unknown. Objective: To evaluate the effects of atorvastatin on dopaminergic and GABAergic neuronal populations in exofocal brain regions in a model of transient occlusion of the middle cerebral artery. Materials and methods: Twenty-eight male eight-week-old Wistar ...
Results No statistically significant differences were found between smokers and non-smokers at the beginning of the observations, except for SCORE (7.13 vs. 3.92, p=0.031) and the disease duration (6.4 vs. 15.9 years, p=0.002). SCORE values, total cholesterol and ICAM-1 serum concentrations decreased during the study in both groups; in smokers: 7.13 vs. 4.44 (p=0.001), 258 vs. 197 mg/dl (p,0.001), 130 vs. 113 pg/ml (p=0.005), respectively, and in non-smokers: 3.29 vs. 3.00 (p=0.069), 261 vs. 213 mg/dl (p=0.012), 132 vs. 108 pg/ml (p=0.018), respectively. After the ATS treatment, CX3CL1 and CCL5 levels dropped significantly only in ever-smoking patients: 1779 vs. 1446 pg/ml (p=0.031) and 1348 vs. 1020 pg/ml (p=0.001), respectively. The ATS treatment had no effect on serum CCL2 and VEGF throughout the observation period.. ...
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A rapid, reversed-phase liquid chromatographic method was developed for the quantitative determination of Atorvastatin calcium, its related substances (12 impurities), and degradation impurities in bulk drugs. The chromatographic separation was achieved on a Zorbax Bonus-RP column by employing a gradient elution with water-acetonitrile-trifluoroacetic acid as the mobile phase in a shorter run time of 25 min. The flow rate was 1.0 mL/min and the detection wavelength was 245 nm. The drug substance was subjected to stress studies such as hydrolysis, oxidation, photolysis, and thermal degradation, and considerable degradation was observed in acidic hydrolysis, oxidative, thermal, and photolytic stress conditions. The formed degradation products were reported and were well-resolved from the Atorvastatin and its related substances. The stressed samples were quantified against a qualified reference standard and the mass balance was found to be close to 99.5% (w/w) when the response of the degradant was
You should not take atorvastatin if you are allergic to it, if you are pregnant or breast-feeding, or if you have liver disease. Stop taking atorvastatin and tell your doctor right away if you become pregnant. Before taking atorvastatin, tell your doctor if you have a thyroid disorder, muscle pain or weakness, a history of liver or kidney disease, or if you drink more than 2 alcoholic beverages daily. In rare cases, atorvastatin can cause a condition that results in the breakdown of skeletal muscle tissue, leading to kidney failure. Call your doctor right away if you have unexplained muscle pain, tenderness, or weakness especially if you also have fever, unusual tiredness, and dark colored urine. Avoid eating foods that are high in fat or cholesterol. Atorvastatin will not be as effective in lowering your cholesterol if you do not follow a cholesterol-lowering diet plan. Avoid drinking alcohol. It can raise triglyceride levels and may increase your risk of liver damage. Atorvastatin is only part ...
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Results: In the TNT (Treating to New Targets) trial, 351 of 3,798 patients randomized to 80 mg of atorvastatin and 308 of 3,797 randomized to 10 mg developed new-onset T2DM (9.24% vs. 8.11%, adjusted hazard ratio [HR]: 1.10, 95% confidence interval [CI]: 0.94 to 1.29, p = 0.226). In the IDEAL (Incremental Decrease in End Points Through Aggressive Lipid Lowering) trial, 239 of 3,737 patients randomized to atorvastatin 80 mg/day and 208 of 3,724 patients randomized to simvastatin 20 mg/day developed new-onset T2DM (6.40% vs. 5.59%, adjusted HR: 1.19, 95% CI: 0.98 to 1.43, p = 0.072). In the SPARCL (Stroke Prevention by Aggressive Reduction in Cholesterol Levels) trial, new-onset T2DM developed in 166 of 1,905 patients randomized to atorvastatin 80 mg/day and in 115 of 1,898 patients in the placebo group (8.71% vs. 6.06%, adjusted HR: 1.37, 95% CI: 1.08 to 1.75, p = 0.011). In each of the 3 trials, baseline fasting blood glucose, body mass index, hypertension, and fasting triglycerides were ...
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Abstract: Wound healing in skin is a complex and dynamic biological process, involving events such as inflammation, proliferation, and remodeling. Recent studies have shown that statins, through pleiotropic effects independent of its lipid lowering action, are new therapeutic options for the treatment of different diseases for their anti-inflammatory and antioxidant actions, on vasodilation and neoangiogenesis and reduction of endothelial dysfunction. The aim of this study was to investigate the effect of atorvastatin in tissue repair after acute injury in healthy animals. The animals were divided into four groups: placebo (P); topical atorvastatin (AT); oral atorvastatin (AO); topical and oral atorvastatin (ATO). Under anesthesia, a lesion in the dorsal region in the animal was made with 8mm punch. The lesions were photographed on days 0, 1, 3, 7, 10 and 14 post-injury and the samples drawn on days 1, 3, 7 and 14 for protein expression analysis of PI3K, Akt, GSK-3, eNOS VEGF, TGF-?, ERK, IL-10, ...
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According to Chang, Test Reload will increase muscle mass (that is Changs central theme with anything he pitches), it can increase a mans libido and improve performance and give men more energy.. How So?. Well, Test Reloads claims are based on the concept of natural testosterone stimulation within the body. This is an important concept to digest. Essentially, Test Reload uses natural herbs and extracts as a way of stimulating the bodys naturally occurring free testosterone. This is important considering pharmaceutical companies sling gels and hormone injections making similar claims. In those cases, however, the body is subjected to artificial or synthetic testosterone. Often the case, the body rejects the foreign substance leaving the patient at risk for side effects and with less cash in their bank account.. In this way, Test Reload wins the first battle in our review. Test Reload does in fact, attempt to regenerate testosterone naturally.. So yes, we are off to a good start.. The bad news ...
Fifty-one children with type 1 diabetes mellitus (DM1) participated in a double blinded, randomized, cross-over pilot study to determine whether 12 weeks of daily atorvastatin (20 mg daily) would reduce arterial stiffness and improve endothelial function. Secondary analysis demonstrated potential reduction of arterial stiffness following atorvastatin therapy (p = 0.06). Additional long-term prospective studies with larger numbers of patients are needed.