Looking for online definition of persistent ductus arteriosus in the Medical Dictionary? persistent ductus arteriosus explanation free. What is persistent ductus arteriosus? Meaning of persistent ductus arteriosus medical term. What does persistent ductus arteriosus mean?
A widened pulse pressure could be a sign of a patent ductus arteriosus in an infant. This is defined as a difference between systolic and diastolic blood pressure of greater than 15 to 25 mmHg, in premature infants and greater than 25 mmHg in term infants [1]. It is thought that diastolic runoff from blood flow shunting across the aorta to the pulmonary artery, through the patent ductus arteriosus, will produce a lower diastolic pressure and therefore widen the pulse pressure in the affected infant [1]. In our retrospective chart review, 116 premature infants were identified with a patent ductus arteriosus and compared to 42 premature infants without a patent ductus arteriosus. The blood pressures obtained were recorded for the first 7 days of life of the patients. Our studies revealed that premature infants with a patent ductus arteriosus had a mean pulse pressure of19 mmHg (p-value 0.129) when compared to infants without a patent ductus arteriosus,16 mmHg, on day 1 of life. The mean pulse pressures
The incidence of symptomatic patent ductus arteriosus (sPDA) in very-low-birth-weight infants has been reported almost exclusively from referral centers. Moreover, the incidence has varied considerably. We prospectively evaluated the incidence and ri
Neonatal Society Abstract: Puddy VF, Amirmansour C, Williams AF, Singer DRJ. 2002. Brain natriuretic peptide concentrations predict haemodynamically significant patent ductus arteriosus in preterm infants.
OBJECTIVE: To report initial experience with a new occlusion device for native and residual patent ductus arteriosus. DESIGN: Descriptive study of consecutive non-randomised patients undergoing a new method of patent ductus arteriosus closure with detachable coils. SETTING: Tertiary centres for paediatric cardiology. PATIENTS: 71 consecutive patients, aged 1.2-22 years, with a patent ductus arteriosus (PDA) underwent elective transcatheter closure. 45 had native PDAs (group A) with a minimum diameter of 1.0 mm-5.0 mm (median 2.0 mm). A further 26 had undergone one or more previous occlusion attempts (group B). INTERVENTIONS: A total of 133 detachable (Cook) spring coils were successfully implanted in 70 patients. The procedure was performed transvenously in 51 patients, retrograde arterially in 13, and by both routes in a further 6 patients. One 5 mm coil migrated but was successfully retrieved. MAIN OUTCOME MEASURES: In group A colour flow Doppler echocardiography showed that complete occlusion ...
TY - JOUR. T1 - Hypotension following patent ductus arteriosus ligation. T2 - The role of adrenal hormones. AU - Clyman, Ronald I.. AU - Wickremasinghe, Andrea. AU - Merritt, T. Allen. AU - Solomon, Tabitha. AU - McNamara, Patrick. AU - Jain, Amish. AU - Singh, Jaideep. AU - Chu, Alison. AU - Noori, Shahab. AU - Sekar, Krishnamurthy. AU - Lavoie, Pascal M.. AU - Attridge, Joshua T.. AU - Swanson, Jonathan R.. AU - Gillam-Krakauer, Maria. AU - Reese, Jeff. AU - Demauro, Sara. AU - Poindexter, Brenda. AU - Aucott, Sue. AU - Satpute, Monique. AU - Fernandez, Erika. AU - Auchus, Richard J.. PY - 2014/6. Y1 - 2014/6. N2 - Objective To test the hypothesis that an impaired adrenal response to stress might play a role in the hypotension that follows patent ductus arteriosus (PDA) ligation. Study design We performed a multicenter study of infants born at ,32 weeks gestation who were about to undergo PDA ligation. Serum adrenal steroids were measured 3 times: before and after a cosyntropin (1.0 μg/kg) ...
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Objective: To study whether indomethacin used in conventional dose for closure of patent ductus arteriosus affects cerebral function measured by Electroencephalograms (EEG) evaluated by quantitative measures. Study design: Seven premature neonates with haemodynamically significant persistent ductus arteriosus were recruited. EEG were recorded before, during and after an intravenous infusion of 0.2 mg/kg indomethacin over 10 min. The EEG was analysed by two methods with different degrees of complexity for the amount of low-activity periods (LAP, "suppressions") as an indicator of affection of cerebral function. Results: Neither of the two methods identified any change in the amount of LAPs in the EEG as compared to before the indomethacin infusion. Conclusion: Indomethacin in conventional dose for closure of patent ductus arteriosus does not affect cerebral function as evaluated by quantitative EEG.. ...
Abstract Objective: To study whether indomethacin used in conventional dose for closure of patent ductus arteriosus affects cerebral function measured by Electroencephalograms (EEG) evaluated by quantitative measures. Study design: Seven premature neonates with haemodynamically significant persistent ductus arteriosus were recruited. EEG were recorded before, during and after an intravenous infusion of 0.2 mg/kg indomethacin over 10 min. The EEG was analysed by two methods with different degrees of complexity for the amount of low-activity periods (LAP, suppressions) as an indicator of affection of cerebral function. Results: Neither of the two methods identified any change in the amount of LAPs in the EEG as compared to before the indomethacin infusion. Conclusion: Indomethacin in conventional dose for closure of patent ductus arteriosus does not affect cerebral function as evaluated by quantitative EEG.
Objective: To study whether indomethacin used in conventional dose for closure of patent ductus arteriosus affects cerebral function measured by Electroencephalograms (EEG) evaluated by quantitative measures. Study design: Seven premature neonates with haemodynamically significant persistent ductus arteriosus were recruited. EEG were recorded before, during and after an intravenous infusion of 0.2 mg/kg indomethacin over 10 min. The EEG was analysed by two methods with different degrees of complexity for the amount of low-activity periods (LAP, "suppressions") as an indicator of affection of cerebral function. Results: Neither of the two methods identified any change in the amount of LAPs in the EEG as compared to before the indomethacin infusion. Conclusion: Indomethacin in conventional dose for closure of patent ductus arteriosus does not affect cerebral function as evaluated by quantitative EEG.. ...
|p|Patent ductus arteriosus (PDA) is the most prevalent cardiovascular defect and is more often seen in females; premature babies are at increased risk. For both sexes, a relationship exists between the risk of this defect occurring and the higher dimensions of the ductus arteriosus. In this study, we examined the relationship between the dimensions of the ductus arteriosus (diameter, length, capacity) and sex. We analyzed a total of 223 fetuses, 108 males and 115 females, ranging in age from four to eight months of intrauterinal life. All fetuses of normal karyotype were obtained from spontaneous abortions. None of the analyzed specimens demonstrated any visible malformations. The increase in the length and diameters of the ductus is linearly related to gestational age. The volumetric growth of this vessel was dependent on fetal age, according to the exponential function. The large number of analyzed specimens allows reliable determination of the ductus arteriosus dimensions in
TY - JOUR. T1 - Increased risk of necrotizing enterocolitis in premature infants with patent ductus arteriosus treated with indomethacin. AU - Grosfeld, Jay L.. AU - Chaet, Mark. AU - Molinari, Francine. AU - Engle, William. AU - Engum, Scott A.. AU - West, Karen W.. AU - Rescorla, Frederick J.. AU - Scherer, L. R.. PY - 1996/9/26. Y1 - 1996/9/26. N2 - Objective: The authors evaluated the risk of necrotizing enterocolitis (NEC) in very low birth weight infants receiving indomethacin (INDO) to close patent ductus arteriosus (PDA). Background Data: Controversy exists regarding the best method of managing very low birth weight infants with PDA and whether to employ medical management using INDO or surgical ligation of the ductus. Methods: Two hundred fifty-two premature infants with symptomatic PDA were given intravenously INDO 0.2 mg/kg every 12 hours x 3 in an attempt to close the ductus. Patients were evaluated for sex, birth weight, gestational age, ductus closure, occurrence of NEC, bowel ...
Continuous infusion versus intermittent bolus doses of indomethacin for patent ductus arteriosus closure in symptomatic preterm infants: Cochrane systematic review answers are found in the Cochrane Abstracts powered by Unbound Medicine. Available for iPhone, iPad, Android, and Web.
TY - JOUR. T1 - Epidemiology, presentation and population genetics of patent ductus arteriosus (PDA) in the Dutch Stabyhoun dog. AU - den Toom, Marjolein L.. AU - Meiling, Agnes E.. AU - Thomas, Rachel E.. AU - Leegwater, Peter A.J.. AU - Heuven, Henri C.M.. PY - 2016/6/13. Y1 - 2016/6/13. N2 - Background: Patent ductus arteriosus (PDA) is one of the most common congenital heart defects in dogs and is considered to be a complex, polygenic threshold trait for which a female sex predisposition has been described. Histological studies in dogs suggest that smooth muscle hypoplasia and asymmetry of the ductus tissue is the major cause of PDA. The Stabyhoun population is small and a predisposition for PDA has been suggested. The aims of this study were to describe the incidence, presentation from a clinical and histopathological perspective, and the population genetics of PDA in the Dutch Stabyhoun population. Results: Forty-six cases were identified between 2000 and 2013. Between 2009 and 2012 the ...
Patent ductus arteriosus with persistent pulmonary artery hypertension after transcatheter closure Jianqi Feng,1,2 Xiangqing Kong,1 Yanhui Sheng,1 Rong Yang1 1Department of Cardiology, the First Affiliated Hospital of Nanjing Medical University, Nanjing, Jiangsu, 2Department of Cardiology, the Second Affiliated Hospital of XuZhou Medical University, XuZhou, Jiangsu, People’s Republic of China Objectives: To observe the change in pulmonary artery systolic pressure (PASP) of patients with persistent pulmonary arterial hypertension (PAH) after patent ductus arteriosus (PDA) occlusion.Background: After occlusion of PDA in patients with PAH, some patients still tend to suffer from persistent PAH.Methods: A chest X-ray, an electrocardiogram, and an echocardiogram were performed on nine patients at 24 hours, 1 and 6 months, and 1 year serially.Results: There was a significant fall (P<0.05) in mean PASP after occlusion (to 59.3±12.7 mmHg). However, the aortic pressure and systemic arterial
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Patent ductus arteriosus is one of the most common congenital heart diseases in the dog. During fetal growth, the ductus arteriosus allows blood to flow from the pulmonary artery into the aorta so that the not-functioning lungs are bypassed. The two main factors that keep the ductus open are ... read more the low oxygen tension in the blood and the high levels of prostaglandin E2 from the placenta. Within minutes to hours after birth, the vessel constricts and closes due to a rise in oxygen tension and the deterioration of placental PGE2. In case of PDA, the ductus arteriosus fails to close after birth. In dogs with PDA, a very typical continuous murmur can be heard on the left heart base. Untreated, PDA will cause death within the first year in one third of all cases. Histopathological, asymmetry and hypoplasia of the ductus muscle is seen. Also, parts of the ductus muscle contain non-contracting aorta like elastic structure. Several dog breeds are predisposed, indicating that there is a ...
Patent ductus arteriosus, or PDA, is a malformation of the heart caused by failure of normal closure of the ductus arteriosus, a direct connection between the aorta and the pulmonary artery which is present and functional during fetal life but which is unnecessary once air-breathing begins. Normally the ductus closes shortly after birth; this process of closure by muscular contraction is reversible for a short time - it can be reopened under hormonal control (prostaglandins). However, within a few weeks it is permanently closed. If the ductus fails to close normally, a loud to-and-fro murmur will result (the so-called machinery murmur). Very young patients with heart or lung disease may need to have the ductus repaired to prevent too much blood from flowing back from the high pressure side of the circulation - the aorta. This lesion also places an older patient at some risk of bacterial endocarditis, so ordinarily the patent ductus is surgically tied off. ...
Patent ductus arteriosus (PDA) is a condition wherein the ductus arteriosus fails to close after birth. Early symptoms are uncommon, but in the first year of life include increased work of breathing and poor weight gain. An uncorrected PDA may lead to congestive heart failure with increasing age. The ductus arteriosus is a fetal blood vessel that closes soon after birth. In a PDA, the vessel does not close and remains "patent" (open), resulting in irregular transmission of blood between the aorta and the pulmonary artery. PDA is common in newborns with persistent respiratory problems such as hypoxia, and has a high occurrence in premature newborns. Premature newborns are more likely to be hypoxic and have PDA due to underdevelopment of the heart and lungs. A PDA allows a portion of the oxygenated blood from the left heart to flow back to the lungs by flowing from the aorta (which has higher pressure) to the pulmonary artery. If this shunt is substantial, the neonate becomes short of breath: ...
The authors report the case of a 14 month old girl with patent ductus arteriosus born from the first, uncomplicated and regulary controlled pregnancy. At 13 months of age a continuous murmur was diagnosed. Doppler echocardiography confirmed patent ductus Botalli and she underwent an operation (because of her age) at the Pediatric Hospital G. Pasquinucci in Massa e Carrara, Italy. During aortography, closure of the ductus arteriosus with large and short infundibulum (dimension 2,0 mm in diameter) was performed with a Gianturco coil (size 5 mm). Procedure was well tolerated and successful. Control Doppler echocardiography revealed normal flow without a shunt ...
The ductus arteriosus is a blood vessel that connects the pulmonary artery and the aorta. In the fetus, it enables blood to bypass the lungs. The fetus does not breathe air, and thus blood does not need to pass through the lungs to be oxygenated. After birth, blood does need to be oxygenated in the lungs, and normally the ductus arteriosus closes quickly, usually within days up to 2 weeks. In patent ductus arteriosus, this connection does not close, allowing some oxygenated blood, intended for the body, to return to the lungs. As a result, the blood vessels in the lungs may be overloaded and the body may not receive enough oxygenated blood.. ...
Patent ductus arteriosus is a medical condition where a blood vessel called the ductus arteriosus does not permanently close after birth. Learn why the ductus arteriosus does not close, why patients with this condition have a wide pulse pressure, and how the disease is treated with surgery and nonsteroidal anti-inflammatory drugs (NSAIDs).
|i|Background:|/i| Preterm infants with a hemodynamically significant patent ductus arteriosus (hsPDA) are at risk for fluctuations in cerebral blood flow, but it is unclear how different
Ligation (clip interruption) of patent ductus arteriosus. This premature baby with operative weight of 600 grams and evidence of persistent pulmonary over
The patency of the ductus arteriosus is very common in extremely premature infants. The increased pulmonary blood flow that results from left-to-right ductal shunting can produce an acute deterioration in lung function and some data suggest that it may increase the risk of chronic lung damage. However, there is no clear evidence that prophylactic or early closure of the patent ductus arteriosus (PDA) results in a clear reduction in bronchopulmonary dysplasia. For this reason, and because of the side effects of the available therapies to close the ductus, there is a wide variation in the approach to the PDA in this population and most clinicians will intervene to close the ductus only in cases of significant shunt with hemodynamic decompensation ...
Patent ductus arteriosus (PDA), where the ductus arteriosus fails to close after birth, is rare in healthy term newborns. As gestational age decreases, the incidence of PDA increases.
The ductus arteriosus is a blood vessel that connects two major arteries before birth and normally closes after a baby is born. If it stays open, the result is a condition called patent ductus arteriosus (PDA).
The ductus arteriosus is a blood vessel that connects two major arteries before birth and normally closes after a baby is born. If it stays open, the result is a condition called patent ductus arteriosus (PDA).
Latest news and research breakthroughs on Patent Ductus Arteriosus. Last updated on Dec 20, 2017 with over 173 News and research items available on the subject. Freely Download - Patent Ductus Arteriosus News Widget - Page 2
Fingerprint Dive into the research topics of Transcatheter closure of the patent ductus arteriosus in adults using the Gianturco coil. Together they form a unique fingerprint. ...
Patent ductus arteriosus (PDA) is a congenital heart defect that occurs when the patent ductus doesnt close. Learn about PDA, including how it affects children and how we perform PDA repair here.
Patent ductus arteriosus (PDA) is presented in 55 to 70% of the preterm infants with a gestational age lower than 30 weeks or a birth weight lower than 1000 grams. PDA has being associated to mortality or morbidity such as ischemic or hemorrhagic cerebral events, necrotising enterocolitis, renal disfunction or poor pulmonary outcome; however, it is not clear whether these are a consequence of the PDA presence, the treatment implemented for closing it, or the immaturity of these population. PDA standard treatment (ST) consists on three doses of indomethacin or ibuprofen (10-5-5mg/kg) given 24 hours apart, being the surgical closure a second line therapeutic option. In spite of ibuprofen has been pointed as the drug of choice for PDA treatment by the last version of the Cochrane review, side effects have been associated to both medication. Standard ibuprofen treatment is based on a clinical trial where the three-dose protocol seemed to be more effective than one-dose scheme for PDA closure; ...
Spontaneous closure of the patent ductus arteriosus in very low birth weight infants following discharge from the neonatal unit ...
Timing of surgical closure of patent ductus arteriosus in preterm neonates?: A retrospective cohort study was conducted on 115 preterm neonates who underwent pa
De Franco P, Chiu T, Kantor N, Garrison R, Miller R. Early surgical closure of patent ductus arteriosus. J Am Osteopath Assoc 1981;80(5):332. doi: https://doi.org/10.7556/jaoa.1981.80.5.332.. Download citation file:. ...
An arteriovenous fistula with aneurysmal dilatation involving branches of both coronary vessels and the pulmonary artery is discussed.. The malformation was limited to the main pulmonary artery and presented a small left-to-right shunt without symptoms.. Ligation of the supplying vessels resulted in the disappearance of a murmur typical of a patent ductus arteriosus.. ...
We used pulsed Doppler ultrasound to determine the flow dynamics of the aorta and patent ductus arteriosus (PDA) in 21 infants (ages 1 to 16 days) with PDA and aortic atresia (n = 15) or aortic stenosis (n = 6). The flow within the PDA was recorded in 19, and was right-to-left during systole in all. There was a diastolic left-to-right PDA shunt in 11 patients with aortic atresia and in three with aortic stenosis, and the shunt was associated with large (3 to 11 mm in diameter) interatrial communications. In two patients with aortic atresia and three with aortic stenosis, however, the diastolic PDA shunt was from right to left, and the interatrial communications were small (0 to 2 mm in diameter). The right-to-left diastolic PDA shunting may be best explained by the relative pulmonary (high with left-sided inflow obstruction and a small interatrial communication) and systemic resistances. All patients with aortic atresia and three with aortic stenosis had retrograde systolic flow in the ...
One type of congenital heart defect is called Patent Ductus Arteriosus (PDA). Although this defect actually affects the vessels around the heart but not the heart itself, it is typically still considered...
... is a heart defect found in the days or weeks after birth. Read on to learn about symptoms, diagnosis, and treatment options.
... is a heart defect found in the days or weeks after birth. Read on to learn about symptoms, diagnosis, and treatment options.
... is a heart defect found in the days or weeks after birth. Read on to learn about symptoms, diagnosis, and treatment options.
Are you looking for a specialist in patent ductus arteriosus (PDA)? Here you will find selected specialists in Germany, Austria or Switzerland.
Patent ductus arteriosus is a common congenital heart disease that can be isolated but is usually present with other co-existing congenital heart anomalies. Learn more about it!
Patent ductus arteriosus is a congenital heart defect in premature babies which may lead to pulmonary hypertension and heart failure.
Learn about symptoms, causes and treatment of patent ductus arteriosus, a common - and treatable - heart defect that occurs shortly after birth.
Pediatric patent ductus arteriosus occurs when there is an opening between the blood vessels that go between the heart and lungs. Learn how this impacts a childs health.
TY - JOUR. T1 - Percutaneous occlusion of patent ductus arteriosus for an elderly patient with refractory congestive heart failure. AU - Shoji, Satoshi. AU - Kanazawa, Hideaki. AU - Yanagisawa, Ryo. AU - Tanaka, Makoto. AU - Fukuoka, Ryoma. AU - Akita, Keitaro. AU - Kimura, Mai. AU - Arai, Takahide. AU - Kawakami, Takashi. AU - Hayashida, Kentaro. AU - Yuasa, Shinsuke. AU - Tsuruta, Hikaru. AU - Itabashi, Yuji. AU - Murata, Mitsushige. AU - Nishiyama, Takahiko. AU - Kohno, Takashi. AU - Maekawa, Yuichiro. AU - Fukuda, Keiichi. PY - 2018/2/1. Y1 - 2018/2/1. UR - http://www.scopus.com/inward/record.url?scp=85052330940&partnerID=8YFLogxK. UR - http://www.scopus.com/inward/citedby.url?scp=85052330940&partnerID=8YFLogxK. U2 - 10.1161/CIRCHEARTFAILURE.117.004764. DO - 10.1161/CIRCHEARTFAILURE.117.004764. M3 - Short survey. C2 - 29367269. AN - SCOPUS:85052330940. VL - 11. JO - Circulation: Heart Failure. JF - Circulation: Heart Failure. SN - 1941-3297. IS - 2. M1 - e004000. ER - ...