Dive into the research topics of An autopsy case of rhino-orbito-cerebral mucormycosis associated with multiple cranial nerve palsy and subsequent subarachnoid hemorrhage. Together they form a unique fingerprint. ...

Humans; Carcinoma, Squamous Cell; Brain Neoplasms; Cranial Nerve Neoplasms; Neoplasm Invasiveness; Cranial Nerve Diseases; Facial Nerve Diseases; Trigeminal Nerve Diseases; Aged; ...

Contributors SG contributed to the diagnosis and management of the case and in writing the first draft of the manuscript. AE contributed to diagnosis, management, the conceptualisation of the report and in writing and critique of the manuscript. SK contributed to the diagnosis and management of the case and in writing the first draft of the manuscript. MM was involved in the discussion of the clinicopathological conference (CPC), writing and critique of the manuscript. MCS was involved in histopathological diagnosis, discussion of the pathology part of the CPC and in writing and critique of the manuscript. KK was involved in histopathological diagnosis, discussion of the pathology part of the CPC and in writing and critique of the manuscript. AG was involved in radiological diagnosis and discussion and writing and critique of the manuscript. Madhavi Tripathi was involved in positron emission tomography-CT interpretation and writing and critique of the manuscript. AK was involved in performing ...

Cranial Neuropathies What are cranial neuropathies? Nerves power your entire body, but those nerves can be damaged by injury or an illness such as diabetes. Neuropathy is a disorder that causes nerve damage and affects your ability to feel and move. Exactly how your body and your movement are affected depends on where in the body the damaged nerves are located. When nerves in the brain or brainstem are affected, it is called cranial neuropathy. The cranial nerves are those that arise directly from your ...

Cranial Nerve Involvement, Large Ears, Otalgia Symptom Checker: Possible causes include Otitis Externa, Mastoiditis, Acoustic Neuroma. Check the full list of possible causes and conditions now! Talk to our Chatbot to narrow down your search.

article{9a3fcc76-7ff7-4e06-9659-b11bead8e138, abstract = {A 52-year-old man developed diplopia, a nasal voice, dysphagia, hoarseness and slight bilateral facial palsies. There was no ataxia, areflexia, limb weakness or sensory involvement. Serum anti-GQ1b IgG antibody was present. Treatment with intravenous immunoglobulin started, and the patient responded with a rapid resolution of symptoms. The diagnosis is consistent with polyneuritis cranialis which is considered to be a Guillain-Barre syndrome variant, a forme fruste, but very rare. The diagnosis can be difficult and a thorough investigation is required. Electrophysiological examination, laboratory evaluations, imaging and cerebrospinal fluid examination are often required in the investigations. Cranial neuropathy can be the presentation of many disorders. Determination of anti-ganglioside antibodies as anti-GQ1b is valuable to the diagnosis, and shows the association with the Guillain-Barre syndrome.}, author = {Edvardsson, Bengt and ...

FINAL DIAGNOSIS:. CMV ventriculo-encephalitis, polyradiculitis, and multiple cranial neuritis. DISCUSSION. CMV has been associated with a variety of central (CNS) and peripheral (PNS) nervous system syndromes in AIDS patients. Among the PNS syndromes, a rapidly progressive lumbosacral polyradiculopathy is the most frequent, occurring in about 1% of AIDS patients (1-6). Cranial nerve involvement in this PNS CMV syndrome has been mentioned only rarely and only in occasional nerves (7,8). Our case is unique in the presence of multiple cranial neuropathies resulting from direct infection of each with CMV. The manifestations of CMV ventricular encephalitis are consistent with this previously described distinct clinical-pathological entity known to occur in HIV-infected patients (9). Another unique aspect in this case is the discrepancy between the minimal MRI findings in the periventricular regions in the presence of diffuse necrotizing ventriculo-encephalitis. A possible explanation for this is the ...

In a prospective study of 271 new patients with nasopharyngeal carcinoma, 36 (13.3%) were found to have cranial nerve involvement. Serous otitis media (SOM) was found in 98 (41.4%) of 237 patients who had undergone complete otologic assessment. The local control of tumor and actuarial survival of three subgroups of patients, namely, 80 patients with SOM only, 11 patients with cranial nerve palsy only, and 18 patients with both SOM and cranial nerve palsy, were analyzed. The local control of tumor was better in patients with SOM alone than in those with cranial nerve palsy alone; those patients with both SOM and cranial nerve involvement had worse local control as well as survival. As SOM is not uncommon in the diagnosis of nasopharyngeal carcinoma, and adult-onset SOM is otherwise distinctly uncommon, this provides a good opportunity for early recognition and, perhaps, better control of nasopharyngeal carcinoma ...

These authors contributed equally to this work DOI : 10.5137/1019-5149.JTN.7876-13.3 Cranial neuropathy is usually idiopathic and familial cases are uncommon. We describe a family with 5 members with cranial neuropathy over 3 generations. All affected patients were women, indicating an X-linked dominant or an autosomal dominant mode of inheritance. Our cases and a review of the literature suggest that familial idiopathic cranial neuropathy is a rare condition which may be related to autosomal dominant vascular disorders (e.g. vascular tortuosity, sclerosis, elongation or extension), small posterior cranial fossas, anatomical variations of the posterior circulation, hypersensitivity of cranial nerves and other abnormalities. Moreover, microvascular decompression is the treatment of choice because vascular compression is the main factor in the pathogenesis. To the best of our knowledge, this is the first report of familial cranial neuropathy in China. Keywords : Cranial nerves, Familial, ...

We observed that most CN palsies had 1 or more MRI findings of intracranial or orbital CN involvement; this may be a reason for the adverse prognostic significance of CN palsies, and patients with CN palsies are determined to be at stage T4.3. Several investigators have reported that the perineural tumor spread in patients with non-nasopharyngeal carcinoma of the head and neck is associated with an increased incidence of recurrence.18-21 However, in our series, MRI-detected CN involvement was not associated with the 3-year LRFS rate in NPC patients with local advanced disease. The primary treatment modality for NPC was radiation therapy, whereas that of other carcinomas of the head and neck was surgery. Lawrence and Cottel reported that postoperative radiotherapy of squamous cell carcinoma with perineural invasion resulted in a much improved survival probability when compared with that observed after conventional surgical excision.20 Radiation therapy (relatively large treatment volume) provided ...

Cranial nerve palsy is a type of muscle malfunction involving at least one of the cranial nerves. Those with cranial nerve palsy...

OBJECTIVES: Functional reorganisation of the motor or sensory cortex has been demonstrated in animals after section of mixed peripheral nerves. Here functional changes in the motor cortex specifically after peripheral motor denervation in humans are investigated. METHODS: Functional MRI (fMRI) was used to study brain activation during a finger flexion-extension task in patients with a late onset, acquired pure motor neuropathy (n=6), contrasting results with those from patients with pure sensory neuropathies (n=4) or healthy controls (n=7). RESULTS: Increases in the extent of activation in the motor cortex both ipsilateral and contralateral to the hand moved were found in the patients with motor neuropathy. The neuroanatomical localisation of the mixed contralateral sensorimotor cortex activation volume was more posterior for the patients with motor neuropathy than for the healthy controls (mean difference, 12 mm, p|0.05). The pure sensory neuropathy group by contrast showed no change in the extent of

The second cranial nerve is called the optic nerve. It sends visual information from the eye to the brain. The third cranial nerve is called the oculomotor nerve. It is involved with eye movement, eyelid movement, and the function of the pupil and lens. The fourth cranial nerve is called the trochlear nerve and the sixth cranial nerve is called the abducens nerve. They each innervate an eye muscle involved in eye movement. The fifth cranial nerve is called the trigeminal nerve. It provides facial sensation (including corneal sensation).. ...

S04.891 is a non-billable code, consider using a code with a higher level of specificity for a diagnosis of injury of other cranial nerves, right side.

Anisocoria in the absence of orbital trauma. Abnormalities in a cranial nerve reflex indicate injury to the brainstem or the neural pathway connecting to the brainstem. Multiple cranial nerve deficits is highly suggestive of brainstem injury. The two most useful cranial nerve reflexes for rapid assessment of brainstem function are the gag reflex and the presence of physiologic nystagmus, otherwise known as the oculocephalic reflex. If these two reflexes are absent it strongly indicates significant brainstem injury and a guarded prognosis.. The segmental spinal reflexes should be evaluated when possible, without excessive manipulation of the patient. Abnormalities in thoracic and/or pelvic limb reflexes will help identify spinal trauma. Cases of brain injury without spinal injury tend to have normal or possibly exaggerated spinal reflexes.. Decerebrate posture: patient has opisthotonos and extensor rigidity of all four limbs. The animal is unresponsive and does not detect deep pain sensation. ...

List of 38 causes for 7th cranial nerve palsy and Arthritis in multiple joints in children and Respiratory muscle paralysis, alternative diagnoses, rare causes, misdiagnoses, patient stories, and much more.

List of causes of 7th cranial nerve palsy and Arthritis in multiple joints in children and Sensations, alternative diagnoses, rare causes, misdiagnoses, patient stories, and much more.

Fingerprint Dive into the research topics of Do patients with neurologically isolated ocular motor cranial nerve palsies require prompt neuroimaging?. Together they form a unique fingerprint. ...

List of causes of Abnormal patellar reflex and Cranial nerve palsy and Face symptoms and Focal seizure and Hyperreflexia and Paraplegia, alternative diagnoses, rare causes, misdiagnoses, patient stories, and much more.

Cranial nerve palsy was found in 5-Minute Clinical Consult. 5-Minute Clinical Consult (5MCC) app and website powered by Unbound Medicine helps you diagnose and manage 900+ medical conditions. Exclusive bonus features include Diagnosaurus DDx, 200 pediatrics topics, and medical news feeds.

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Publication Year and Month: 1997 05. Abstract: Here we report a case of a 56-year-old male with post-poliomyelitis muscular atrophy (PPMA), who presented with cranial nerve signs and widespread atrophy of the extremities. He had suffered from poliomyelitis at the age of 2 years. After recovery from the acute stage, the paralysis remained in his left arm. He noticed muscle weakness of the right upper and lower extremities at the age of 45 years and the muscle atrophy progressed to his arms, hip and thigh at the age of 55 years. Neurological examination revealed muscle atrophy of the neck and disturbance of left V, VIII, IX, X and bilateral XI cranial nerves. We diagnosed this case as PPMA from his history and electromyographic and muscle biopsy findings which suggested chronic denervation. Among the 21 PPMA cases in the past in which the acute poliomyelitis had resulted in paralysis of the only one limb, ours was the only case that had muscle atrophy of all the limbs. Cranial nerve involvement is ...

The present invention provides a composition for preventing, alleviating, or treating cranial nerve diseases caused by dopamine deficiency, the composition containing triiodidethyronine and tyroxine, or a salt thereof. According to the present invention, the triiodidethyronine and tyroxine, or a salt thereof significantly promote the expression and proliferation of dopamine neurons, thereby exhibiting excellent effects in the prevention and treatment of cranial nerve diseases caused by dopamine deficiency, such as Parkinson´s disease.

Rare: Occurs in only 5% of sarcoidosis Usually presents in pt with known sarcoid Usually other neuro manifestations also: Optic neuropathy / other cranial nerve deficits Encephalopathy Aseptic meningitis Motor weakness Peripheral neuropathy Consider as cause of isolated CN7 Palsy if: Recurrent palsies No improvement at all in facial weakness after 3 mos. Send to…

The MR changes that accompany cranial and peripheral motor denervation are well described and include asymmetrical decrease in affected muscle volume, fatty infiltration of the involved muscle group, and variable signal intensity changes, including both T2 prolongation and postcontrast enhancement (2, 3, 5, 7-9). While CT is able to demonstrate only the chronic changes of atrophy and fatty replacement (1), the superior soft tissue contrast of MR facilitates the depiction of the progressive evolution from an acute phase to a subacute and then to a chronic phase that denervated muscle may undergo. In some patients with peripheral neuropathy and either spontaneous resolution of paralysis or surgical nerve grafting, acute or subacute changes have been shown to be reversible on MR imaging, corroborating clinical evidence of reinnervation (7, 9). To our knowledge, this reversibility has not been demonstrated in the setting of cranial neuropathy, presumably because the processes that lead to MR ...

TY - JOUR. T1 - Response of extra-axial tumors to stereotactically implanted high-activity 125l seeds. AU - Patil, Arun Angelo. AU - Kumar, Pradeep. AU - Leibrocka, Lyal G.. PY - 1995/1/1. Y1 - 1995/1/1. N2 - 125I seeds can deliver a very high dose of radiation (100-500 Gy) to a well-circumscribed area over their average life of 87 days, which enable them to destroy slow-growing extra-axial tumors after permanent implantation. Stereotactic implantation of 125I seeds was performed in 26 patients with extra-axial tumors, with a median follow-up of 32 months. No acute morbidity or mortality resulted from the procedure. Cranial nerve involvement in 5 patients was the only delayed complication. One patient developed radionecrosis outside the range of 125I dose distribution, which was felt to be due to the hyperfractionated external radiation the patient had received prior to 125I seed placement. All 26 patients showed tumor regression. The results indicate that this form of treatment is relatively ...

The act of swallowing is complex. Approximately 40 oral and pharyngeal (mouth and throat) muscles and multiple cranial nerves work together to accomplish two critical, life sustaining actions: breathing and swallowing. Weak oropharyngeal muscles can prevent effective and efficient transport of food, liquids and medication causing them to flow into an unprotected airway, also known as aspiration. Swallowing disorders, also known as dysphagia, are highly prevalent in stroke patients and are associated with increased mortality and morbidity, dehydration, pulmonary complications, and reduced rehabilitation potential. Therefore, it is critical to ensure Medicare patients have access to safe, effective therapies to improve their swallowing function, prevent further complications and continue to advance their rehabilitation ...

Diagnosis Code S04.891D information, including descriptions, synonyms, code edits, diagnostic related groups, ICD-9 conversion and references to the diseases index.

A Canadian non-profit registered charitable organization for those who suffer from TN or any other facial pain. Find disease info, a newsletter and links. ...

Germinomas typically arise in regions adjacent to the third ventricle, such as the pineal body and neurohypophysis, but they may be present also in the basal ganglia. Germinomas originating in the cerebellum are quite rare [5-11] (see Additional file 2: Table S1); in one study, only three of 153 intracranial GCTs were located in the cerebellum [12]. It is not difficult to diagnose intracranial GCTs when they have a typical location and radiographic features. However, an intracranial GCT located in the basal ganglia often is difficult to detect because enhancement is usually slight or absent [2]. Recent reports have emphasized the effectiveness of SWI in early detection of basal ganglia GCTs [13]. SWI can be used to detect blood products and accumulation of biologic metal, thereby providing additional information in evaluation of brain tumors. SWI may be useful also in the detection of intracranial GCTs, because this type of tumor is characterized by a high incidence of intra-tumor micro bleeds. ...

Viewed posteriorly the right kidney has its upper edge opposite the 11th dorsal spine and the lower edge of the 11th rib. Its lower edge is ...

21-year-old female with an acute onset of a unilateral sixth cranial nerve deficit. Brain MRI showed at least a dozen white matter lesions, one of them contrast-enhancing. Sensory evoked potentials showed a bilateral increase in latency. Multiple sclerosis was diagnosed, and the patient was treated by corticosteroids, followed by improvement of symptoms. The first follow-up revealed no focal neurological signs with a history of a transitory unilateral lower limb hyposthenia. On the second visit, a horizonto-rotatory nystagmus and irregular paraesthesia in the right toes are documented. ...

Lower cranial nerve (IX-XII) palsy is a rare condition with numerous causes, usually non-traumatic. In the literature it has been described only a few times after trauma, mostly accompanied by a fracture of the occipital condyle. Although these types of fractures have rarely been reported one could suspect they have been under-diagnosed. During the past decade they have been seen more frequently, most probably due to increased use of CT- and MRI-scanning. The purpose of this review is to increase the awareness of complications following injuries in the craniocervical region. We based this article on a retrospective review of the medical record of a 24-year old woman admitted to our trauma center after being involved in a car accident and a review of the literature on occipital condyle fractures associated with lower cranial nerve palsy. The multitraumatized patient had suffered a dislocated occipital condyle fracture. Months later she was diagnosed with palsy to cranial nerve IX-XII. Literature review

저자들은 경정맥공의 종양에 대해서 종전(2000년 이전)에는 종양의 크기와 상관없이 CWD 방법으로 수술하였으나, 최근(2000년 이후) 수술한 예들에서는 종양의 크기 및 위치, 종류에 따라 유양돌기 절제술의 방법과 안면 신경 전위의 위치를 다양하게 하여 가능한 한 기능적 보존을 시도하였다. 종전에 수술한 예는 JFS가 4예, 후두개저의 뇌수막종이 1예로서 종양의 평균 크기는 23.2 mm로 비교적 작은 크기의 종양이었지만 모두 CWD-1G 방법을 사용하여 종양에 접근하였다. 2000년 이후에 시행된 최근 증례로는 JFS가 5예, 경정맥 사구종이 2예, 연골육종이 1예가 있다. JFS 5예 중 3예는 CWU-2G를 시행하였고 종양의 평균 크기는 20.0 mm였으며, CWD-1G를 시행한 2예는 평균 크기 43.9 mm로 작은 크기의 종양에 대해서는 외이도 후벽을 보전하고 안면 신경의 조작을 최소화하려고 ...

We describe a rare neurological presentation of internal jugular vein thrombosis induced by central venous catheter placement in a patient with cancer. A 71-year-old man gave a 3-week history of dysphagia and dysarthria with left-sided neck pain and headache. He was receiving chemotherapy for appendiceal adenocarcinoma. On examination, he had left 9th-12th cranial neuropathies, manifesting as voice hoarseness, decreased palatal movement, absent gag reflex, weakness of scapular elevation and left-sided tongue wasting. CT scan of neck showed the left subclavian central venous catheter tip was in the left internal jugular vein. Skull base MRI showed thrombus within the left jugular foramen extending intracranially. We diagnosed Collet-Sicard syndrome secondary to thrombosis in the sigmoid-jugular venous complex. His headache and neck pain resolved 2 days after removing the catheter and starting anticoagulation. Collet-Sicard syndrome is an unusual syndrome of lower cranial nerve palsies, often ...

Although the cranial nerves and their sensory and. of the brainstem showing the cranial nerves.Which cranial nerve carries sensory fibers from taste receptors of.This is an article introducing the 12 cranial nerves. this nerve governs the ocular and sensory functions.. The Cranial Nerves (Organization of the Central Nervous. motor neuron lesion of this cranial nerve (described in the following.The patient complains of decreased sense of taste (3 cranial nerves).Nerve - Cranial Nerve 9,10 The functions of the. 2004 - 08 Cranial Nerves.The olfactory nerve is a special sensory cranial nerve that ...

MalaCards based summary : Schwannoma of Twelfth Cranial Nerve, also known as schwannoma of the twelfth cranial nerve, is related to neurilemmoma and plexiform schwannoma. An important gene associated with Schwannoma of Twelfth Cranial Nerve is NF2 (Neurofibromin 2). Affiliated tissues include 12th cranial nerve, tongue and thymus, and related phenotypes are Decreased cell migration and Increased cell migration ...

We report a 55-year-old man who presented with a moderately severe community acquired pneumonia due to Chlamydia psittaci, probably acquired from budgerigars. One of his presenting symptoms was diplopia, secondary to bilateral 4th cranial nerve palsies, which persisted for some months. The literature on neurological complications of pneumonia is reviewed.

List of 28 causes for 7th cranial nerve palsy and Beaus lines in children and Beaus lines in children - toenail and Paraesthesia of the lower extremity, alternative diagnoses, rare causes, misdiagnoses, patient stories, and much more.

Fingerprint Dive into the research topics of Cranial neuropathy and severe pain due to early disseminated Borrelia burgdorferi infection. Together they form a unique fingerprint. ...

Heres Everything You Need To Know About The Cranial Nerves - How Many Cranial Nerves Are There And The Cranial Nerves Function. How To Remember Cranial Nerves, In Order And Labeled. Learn About Brainstem

Definition of Cranial nerve nucleus in the Legal Dictionary - by Free online English dictionary and encyclopedia. What is Cranial nerve nucleus? Meaning of Cranial nerve nucleus as a legal term. What does Cranial nerve nucleus mean in law?

Nerve conduction defects (weakness/paralysis of limbs, loss of reflexes, tingling sensations in the extremities); severe headaches; stiff neck; meningitis; dizziness; fainting; cranial nerve involvement (change in smell/taste; difficulty chewing, swallowing, or speaking; hoarseness or vocal cord problems; facial paralysis known as Bells palsy; drooping shoulders; inability to turn head; double vision); abnormal brain waves or seizures; sleep disorders; cognitive changes (memory problems, confusion, disorientation, decreased concentration); behavioral changes (depression, personality changes, panic attacks); mental illness (sudden onset with no previous history of mental illness).. Heart and Blood Vessels ...

Onset of first symptom has been reported between 1-12 years, with a mean age of onset at 8 years. Clinical course can be divided into early (, 6 yrs age, predominance of respiratory symptoms) and late course (6-20 years of age, predominance of motor symptoms on superior limbs). Progression to involve other cranial nerve muscles occurs over a period of months or years. In the Gomez review facial nerve was affected in all cases while hypoglossal nerve was involved in all except one case. Other cranial nerves involved were vagus, trigeminal, spinal accessory nerve, abducent, occulomotor and glossopharyngeal in this order. Corticospinal tract signs were found in 2 of the 14 patients. The disease may progress to patients death in a period as short as 9 months or may have a slow evolution or may show plateaus. Post mortem examination of cases have found depletion of nerve cells in the nuclei of cranial nerves. The histologic alterations found in patient with Fazio-Londe disease were identical to ...

RESULTS: Primary CNSL was typically localized supratentorially (63%), with multiple (59%) or infiltrative (36%) lesions showing diffusion restriction (98%), often (87%) reaching the brain surface. In approximately 50% of patients, meningeal, ependymal or cranial nerve involvement was found. We detected significant differences in enhancement patterns between immunocompromised and immunocompetent patients; non-homogenous enhancement present in 50% of immunocompromised patients. We did not find any significant differences in MRI appearance between primary and secondary CNSL. Regression was evident after corticosteroid treatment in 52% of patients; however, in 16% of cases overall progression was observed ...

The mean follow-up duration was 84 months (median 75.5 months, range 24-216 months). In 118 patients (86%), the tumor volume was unchanged or had decreased at last follow-up. Kaplan-Meier analysis demonstrated radiographic progression-free survival at 5 and 10 years to be 95.4% and 69%, respectively. Fourteen patients (10%) developed new cranial nerve palsies following GKS. Factors associated with tumor control included younger age, a higher isodose, and smaller tumor volume. A longer follow-up duration was associated with either a decrease or increase in tumor volume. Fourteen patients (10%) experienced new or worsening cranial nerve deficits after treatment. Factors associated with this occurrence were larger pretreatment tumor volume, lower peripheral radiation dose, lower maximum dose, tumor progression, and longer follow-up. ...

Case Reports in Infectious Diseases is a peer-reviewed, Open Access journal that publishes case reports related to infectious diseases of bacterial, viral and parasitic origin.

Cranial Nerve 1- Olfaction This patient has difficulty identifying the smells presented. Loss of smell is anosmia. The most common cause is a cold (as in this patient) or nasal allergies. Other causes include trauma or a meningioma affecting the olfactory tracts. Anosmia is also seen in Kallman syndrome because of agenesis of the olfactory bulbs. Cranial Nerve 2- Visual acuity This patientâs visual acuity is being tested with a Rosenbaum chart. First the left eye is tested, then the right eye. He is tested with his glasses on so this represents corrected visual acuity. He has 20/70 vision in the left eye and 20/40 in the right. His decreased visual acuity is from optic nerve damage. Cranial Nerve II- Visual field The patients visual fields are being tested with gross confrontation. A right sided visual field deficit for both eyes is shown. This is a right hemianopia from a lesion behind the optic chiasm involving the left optic tract, radiation or striate cortex. Cranial Nerve II- Fundoscopy ...

Multiple cranial nerves may be involved (nerves V, VI, VII, IX, X, and XII can all be affected) and lead to orofacial malformations, abnormalities of tongue coordination, swallowing (bulbar muscle weakness), and drooling. Speech abnormalities are common. High risk for corneal abrasions. Usually the affected individuals are mentally normal. However, there is a 10% incidence of mild mental retardation. Occasionally peripheral neuropathy and pectoral muscle deficiency. Abnormalities of ventilation (central hypoventilation and idiopathic tachypnea) presumably result from brainstem abnormalities. Craniofacial features include micrognathia ...

The problem of diagnosing vasculitic neuropathy is discussed based on case reports of two patients with Wegeners granulomatosis. One patient developed de novo 6(th) nerve palsy as an isolated relapse manifestation and the second patient a sequence o

Overall tumor control was achieved in 93.4% of patients at last follow-up; actuarial tumor control was 98%, 95%, 91%, and 85% at 3, 5, 8, and 10 years postradiosurgery, respectively. Smaller adenoma volume (OR 1.08 [95% CI 1.02-1.13], p = 0.006) and absence of suprasellar extension (OR 2.10 [95% CI 0.96-4.61], p = 0.064) were associated with progression-free tumor survival. New or worsened hypopituitarism after radiosurgery was noted in 21% of patients, with thyroid and cortisol deficiencies reported as the most common postradiosurgery endocrinopathies. History of prior radiation therapy and greater tumor margin doses were predictive of new or worsening endocrinopathy after GKS. New or progressive cranial nerve deficits were noted in 9% of patients; 6.6% had worsening or new onset optic nerve dysfunction. In multivariate analysis, decreasing age, increasing volume, history of prior radiation therapy, and history of prior pituitary axis deficiency were predictive of new or worsening cranial nerve ...

Aims and Objective: To evaluate the clinical and pathological characteristics of nasopharyngeal carcinoma (NPC) patients seen in the Radiotherapy and Oncology department, Ahmadu Bello University Teaching Hospital, Nigeria. Materials and Methods: Between January 2006 and December 2010, 45 patients with histologically confirmed NPC were seen and evaluated irrespective of age, co-morbidity and performance status. Patients folders were reviewed retrospectively with a structured pro forma. Data were analyzed using Epi Info software and results presented in simple tables. Results: A total of 45 patients had NPC accounting for 2%. The mean age was 42 years (range 15-75 years). The sex ratio was M:F = 2.2:1. 21 of the patients were from North-West geopolitical zone. Hausa-Fulani was the predominant ethnic group in 23 patients. At presentation, 41 had neck mass followed by nasal blockage in 34, cranial nerve deficits in 27 and epistaxis in 25 (55.6%) patients. The commonest cranial nerves affected were ...

Introduction Examination of the cranial nerves allows one to view the brainstem all the way from its rostral to caudal extent. The brainstem can be divided into three levels, the midbrain, the pons and the medulla. The cranial nerves for each of these are: 2 for the midbrain (CN 3 & 4), 4 for the pons (CN 5-8), and 4 for the medulla (CN 9-12). It is important to remember that cranial nerves never cross (except for one exception, the 4th CN) and clinical findings are always on the same side as the cranial nerve involved. Cranial nerve findings when combined with long tract findings (corticospinal and somatosensory) are powerful for localizing lesions in the brainstem. Cranial Nerve 1 Olfaction is the only sensory modality with direct access to cerebral cortex without going through the thalamus. The olfactory tracts project mainly to the uncus of the temporal lobes. Cranial Nerve 2 This cranial nerve has important localizing value because of its x axis course from the eye to the occipital ...

Do You Have Cranial Nerve Vii Diseases? Join friendly people sharing true stories in the I Have Cranial Nerve VII Diseases group. Find support forums, advice and chat with groups who share this life experience. Cranial Nerve VII Diseases anonymous su...

The next time youre trying to remember the locations and functions of the cranial nerves, picture this drawing. All twelve cranial nerves are represented, though some may be a little harder to spot than others. For example, the shoulders are formed by the number 11 because cranial nerve XI controls neck and shoulder movement. If you immediately recognize that the sides of the face and the top of the head are formed by the number 7, youre well on your way to using this memory device.. Tags: nerfs craniensneurologieneurology. ...

Diabetes accounts for approximately one-third of cases of neovascular glaucoma. Neovascular glaucoma is a rare type of glaucoma that often results in visual loss. Neovascular glaucoma can occur when new blood vessels grow on the iris, closing off fluid flow in the eye and raising the eye pressure. It can be very difficult to treat, which is why those with diabetes should get their eyes examined on a regular basis.. In addition, people with diabetes are 60 percent more likely to develop cataracts, which is a serious condition that causes the eyes lens to cloud and interfere with normal vision. Cataracts is a condition that tends to develop at an earlier age in people with diabetes.. Those with diabetes are also more at risk for developing microvascular cranial nerve palsies. Microvascular cranial nerve palsies involve the small bloods vessels that affect the muscles that move the eyes. Symptoms of this condition include not being about to move the eyes in different directions, causing double ...

Brainstem cavernous malformations (CMs) pose significant challenges to neurosurgeons because of their deep locations and high surgical risks. Most patients with brainstem CMs present with sudden-onset cranial nerve deficits or ataxia, but uncommonly patients can present in extremis from an acute hemorrhage, requiring surgical intervention. However, the timing of surgery for brainstem CMs has been a controversial topic. Although many authors propose delaying surgery into the subacute phase, some patients may not tolerate waiting until surgery. To the best of the authors knowledge, emergency surgery after a brainstem CM hemorrhage has not been described. In cases of rapidly progressive neurological deterioration, emergency resection may often be the only option. In this retrospectively reviewed small series of patients, the authors report favorable outcomes after emergency surgery for resection of brainstem CMs. ...

EHV-1 usually manifests as a respiratory disease, but can occasionally mutate to a form that affects the nervous system. Infected horses may develop symptoms such as weakness or paralysis of the hind legs giving rise to the dog-sitting position, loss of tail and anal tone, inability to urinate or defecate, urine dribbling, cranial nerve deficits, recumbency, and death. No specific treatment is available for EHV-1, but general supportive therapy and care can aid recovery of affected horses. Anti-inflammatory agents may be helpful in minimizing damage to the spinal cord. The human drug acyclovir was used in a recent EHV-1 outbreak in Ohio, and efficacy of this treatment seems promising. ...

The disease in these patients may be newly acquired or a reactivation. It may be characterized as follows: CNS toxoplasmosis occurs in 50% of patients - Seizure, dysequilibrium, cranial nerve deficit... more

The tumour is slow growing and symptoms are often present for some time prior to diagnosis. Symptoms relate to the site; sacral tumours present with pain and sacral neuropathy; clival tumours with headache and cranial neuropathies (usually ocular), and spine tumours with pain and neuropathy.. ...

Clinic staff had diluted a 100-?g vial of pure neurotoxin with diluent and drew up the resulting solution into syringes for clinical use. The physician working at the clinic administered 4 case-patients (including himself) 4 to 6 injections of this toxin solution in the facial area. All the patients eventually reported symptoms of progressive weakness and cranial neuropathies (abnormality of the nerves that control a number of functions, including movement of the facial muscles and swallowing), with two patients experiencing shortness of breath.. The researchers report the patients may have received doses 2,857 times the estimated human lethal dose by injection. Pretreatment serum toxin levels in 3 of the 4 case-patients were equivalent to 21 to 43 times the estimated human lethal dose; pretreatment serum from the fourth epidemiologically linked case-patient was not available. A 100-?g vial of toxin taken from the same manufacturers lot as toxin administered to the case-patients contained a ...

NEURONAL NUCLEAR ANTIBODIES. Antineuronal Nuclear Antibody-Type 1 (ANNA-1). ,1:240. Antineuronal Nuclear Antibody-Type 2 (ANNA-2). ,1:240. Antineuronal Nuclear Antibody-Type 3 (ANNA-3). ,1:240. Anti-Glial/Neuronal Nuclear Antibody-Type 1 (AGNA-1). ,1:240. NEURONAL AND MUSCLE CYTOPLASMIC ANTIBODIES. Purkinje Cell Cytoplasmic Antibody, Type 1 (PCA-1). ,1:240. Purkinje Cell Cytoplasmic Antibody, Type 2 (PCA-2). ,1:240. Purkinje Cell Cytoplasmic Antibody, Type Tr (PCA-Tr). ,1:240. Amphiphysin Antibody. ,1:240. CRMP-5-IgG. ,1:240. Note: Titers lower than 1:240 are detectable by recombinant CRMP-5 Western blot analysis. CRMP-5 Western blot analysis will be done on request on stored serum (held 4 weeks). This supplemental testing is recommended in cases of chorea, vision loss, cranial neuropathy, and myelopathy. Call the Neuroimmunology Laboratory at 800-533-1710 or 507-266-5700 to request CRMP-5 Western blot.. Neuron-restricted patterns of IgG staining that do not fulfill criteria for amphiphysin, ...

Since the pre-IMRT era, re-irradiation has been shown to be effective in non-metastatic, recurrent NPC (rNPC) patients after primary RT.45 46 47 With its introduction, IMRT has quickly emerged as the radiation modality of choice for rNPC as well, with or without the use of chemotherapy. Its efficacy has been established in various studies, with documented long-term OS rates ranging from 45% to 65%.48 49 50 51 52 53 54 55 56 Yet, most of the patients in those studies were treated with conventional 2D-RT in the pre-IMRT era. In a recent study conducted by Kong et al,56 77 patients received salvage IMRT for rNPC after a definitive course of primary IMRT. While the median OS and PFS were 37.0 and 20.5 months, respectively, of particular note is the re-irradiation toxicity. Of 34 patients, 18 died from treatment-induced severe adverse effects without evidence of disease progression during the study, including mucosal necrosis, temporal lobe necrosis, and cranial neuropathy,56 reflecting the ...

Now, take a look at what I call cranial nerve maps. These are icons of cranial nerves, their branches, what they supply and what foramina they use. Sensory is yellow and motor is red. Solid red is for skeletal muscle. Dashed red is for parasympathetic fibers. You sort of have to memorize which nerves have motor or sensory or both modalities, then consider what part of the head and neck is involved with each nerve. For example, when you look at the olfactory road map, you will see that it is yellow which means it is purely sensory. Then you consider what kind of sensation of picked up in the periphery which, in this case, is smell. The smell impulses then go back to the central nervous system. If there is red included in the map, that means that something will receive efferent fibers and will either contract or secrete. The impulse starts out in the central nervous system and passes out to a muscle or gland. ...

Ok then. Lets get back to our slide and make sure that you are comfortable and familiar with what we see here in this slide and that you know what the 12 cranial nerves are and the 10 that connect to the brain stem in particular for this lesson. Now notice that in this figure theres a color code and the color code is meant. To help you understand which nerves are purely sensory. Which are purely motor. And then, which nerves. Like, most of our spinal nerves have a mixture of sensory and motor axons within them. Well, that may seem, perhaps, surprising that there would be nerves that are mixed for sensation, and. Motor output but that is the case for our spinal nerves and it is the case, as you can see by the abundance of green nerves here, for several of the cranial nerves. So that suggests that there must be some complex relationship between the brain stem and the nerves. And often that is the case. So what I want to help you with next is to understand, how do these nerves connect up with the ...

Ok then. Lets get back to our slide and make sure that you are comfortable and familiar with what we see here in this slide and that you know what the 12 cranial nerves are and the 10 that connect to the brain stem in particular for this lesson. Now notice that in this figure theres a color code and the color code is meant. To help you understand which nerves are purely sensory. Which are purely motor. And then, which nerves. Like, most of our spinal nerves have a mixture of sensory and motor axons within them. Well, that may seem, perhaps, surprising that there would be nerves that are mixed for sensation, and. Motor output but that is the case for our spinal nerves and it is the case, as you can see by the abundance of green nerves here, for several of the cranial nerves. So that suggests that there must be some complex relationship between the brain stem and the nerves. And often that is the case. So what I want to help you with next is to understand, how do these nerves connect up with the ...

Free pdf of cranial nerves, with emphasis on those that relate to voice & swallowing. Patients dysphagia need a full exam of their cranial nerves.

Cranial Nerve Examination - Free download as Word Doc (.doc), PDF File (.pdf), Text File (.txt) or read online for free. Guide to cranial nerve examination

Cranial nerve damage can cause sensory, motor function and parasympathetic abnormalities, depending on which of the 12 cranial nerves are affected. Read this informative article to learn about damage results and treatment options.

The cranial nerves originate in the brain and have power over some of the most important neurological functions of the body. Cranial nerves relay information between the brain and parts of the body, primarily to and from regions of the head and neck.

The cranial nerves are a set of 12 nerves which emerge directly from the brain. The names of the cranial nerves relate to their function and they are also numerically identified in roman numerals I-XII by their specific location of the brain and by the order in which they exit the cranium.

There are many cranial nerve mnemonics that can be memorable and rude/lewd. Either way, they can be helpful for remembering the names of the twelve cranial nerves, as well as remembering which nerves are sensory, motor, or both. Remembering cran...

Upledger Institute UK Cranial Nerves Wallchart [cnchart] - This beautiful wall-sized chart designed by Jean-Pierre Barral, D.O. and Alain Croibier, D.O. allows you to see the cranial nerves you can access in treatment in vivid detail. The full-color illustration highlights the nerves, their exit points through the

Can you find the cranial nerves in this puzzle? Print out this page, then circle all cranial nerve names that you find. The words can be up, down or backwards. There are also a few cranial nerves MISSING from this puzzle. Do you know which ones are missing? For more information of the cranial nerves, go the Cranial Nerve Page. Here is the puzzle: ...

Buy Human Cranial Nerve Skull 4-D Vision Anatomy Model Kit at Entertainment Earth. Mint Condition Guaranteed. FREE SHIPPING on eligible purchases. Shop now!

by SYP@dmin , Sep 1, 2017 , cranial nerves, dysphagia, patient care, Podcast. Todays Swallow Your Pride guest is Tiffani Wallace! Tiffani is one of my most favorite people in this world, shes board certified in swallowing, a blogger, author, presenter, and speaker. She created the Dysphagia Ramblings blog, DysphagiaTherapy Group ...

Study Flashcards On [visual] Cranial Nerves & Brainstem at Cram.com. Quickly memorize the terms, phrases and much more. Cram.com makes it easy to get the grade you want!

Study Flashcards On Cranial Nerves, Brainstem Location and Nuclei at Cram.com. Quickly memorize the terms, phrases and much more. Cram.com makes it easy to get the grade you want!

We wouldnt be able to talk, taste, chew, or swallow without the cranial nerves of our face and mouth. Find out how these nerves help us move our...