Looking for Collagen-vascular disease? Find out information about Collagen-vascular disease. see autoimmune disease autoimmune disease, any of a number of abnormal conditions caused when the body produces antibodies to its own substances. Explanation of Collagen-vascular disease
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Nonetheless, due to limited space we do ask that you invite no collagen vascular disease and pregnancy than 2 collaegn. With my 2nd youngster, I keep in mind getting that crampy feeling right after and it lasted for awhile. Now that a variety of hCG or human Chorionic Gonadotropin hormone is secreted within the physique extra meals aversions, sensitivity to odor, fatigue and constipation may be expected. As I discussed, these guys do not have these issues. Collahen involuntary leaking could occur when coughing, sneezing and collagen vascular disease and pregnancy laughing. As mentioned earlier, its not unusual for a woman to spot during her first 12 weeks of being pregnant. When crucial, I embrace further postpartum visits to insure that diseasd gets off to a terrific begin. Oxytocin itself may cause uterine contractions and thus Collagen vascular disease and pregnancy can cross-react with oxytocin receptors and trigger contractions. A prenatal multivitamin doesnt replace a healthy diet. Cite ...
Alport syndrome is a hereditary type IV collagen disease leading to progressive renal fibrosis, hearing loss and ocular changes. End stage renal failure usually develops during adolescence. COL4A3-/- mice serve as an animal model for progressive renal scarring in Alport syndrome. The present study evaluates the role of Discoidin Domain Receptor 1 (DDR1) in cell-matrix interaction involved in pathogenesis of Alport syndrome including renal inflammation and fibrosis. DDR1/COL4A3 Double-knockouts were compared to COL4A3-/- mice with 50% or 100% expression of DDR1, wildtype controls and to DDR1-/- COL4A3+/+ controls for over 6years. Double-knockouts lived 47% longer, mice with 50% DDR1 lived 29% longer and showed improved renal function (reduction in proteinuria and blood urea nitrogen) compared to animals with 100% DDR1 expression. Loss of DDR1 reduced proinflammatory, profibrotic cells via signaling of TGFbeta, CTGF, NFkappaB and IL-6 and decreased deposition of extracellular matrix. ...
title:Socio Demographic Profile and Clinical Presentation of Collagen Vascular Disease with Pulmonary Symptoms: A Descriptive Cross Sectional Study. Author:Parul Vadgama, Mehul Bhavsar, Kusum V Shah, Rajesh Solanki, Ramakant Dixit, Jignesh Vaishnani. Keywords:Collagen vascular disease, pulmonary symptoms, sceroderma, rheumatoid arthritis. Type:Original Article. Abstract:Introduction: Collagen Vascular Disease presented with varied systemic symptoms including pulmonary symptoms, commonly breathless on exertion; dry cough; cough with expectoration; chest pain; and hemoptysis. The current study was conducted to know socio demographic profile and clinical presentation of patient coming with Collagen Vascular Disease having pulmonary symptoms. Methodology: This was a descriptive cross sectional study conducted among Collagen Vascular Disease patient coming with pulmonary symptoms in outpatient department. Results: Among the 50 patients 40% patients were having SC followed by SLE (30%). Most common ...
Question - I have a past history of collagen vascular disease and leukopenia. Is swollen bulging eye and pain behind the ear a symptom of connective tissue disease and autoimmune thyroid ?. Ask a Doctor about Thyroid, Ask a Hematologist
List of causes of Collagen vascular diseases causing fever and Dry scalp in children and Food symptoms and Heart symptoms and Metacarpophalangeal joint sensitive, alternative diagnoses, rare causes, misdiagnoses, patient stories, and much more.
List of causes of Collagen vascular diseases causing fever and Difficulty rising from a chair and Food symptoms and Heart symptoms and Metacarpophalangeal joint sensitive, alternative diagnoses, rare causes, misdiagnoses, patient stories, and much more.
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Wet-lab validated real-time PCR primer assays for your biological pathway of interest. Select your gene target of interest using an interactive pathway map, and select your plate.
B. Often the diagnosis is obscured procsar of the severe degree proscar in thailand kidney destruction. Pedrotti, is associated with rheumatoid arthritis (or other collagen disease) in approximately Prтscar of patients (see section on Scleritis in Chapter 8), and approximately 45 have a known systemic condition.
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Prednisolone (Predonema). Prednisolone is used for treating allergies, arthritis, breathing problems (eg, asthma), certain blood disorders, collagen diseases (eg, lupus), certain eye diseases (eg, keratitis), cancer (eg, leukemia), endocrine problems (eg, adrenocortical insufficiency), intestinal problems (eg, ulcerative colitis), swelling due to certain conditions, or skin conditions (eg, psoriasis).. Other names for this medication. Adelcort, Adelone, Aersolin d, Ak-pred, Alertine, Alpicort, Apicort, Aprednislon, Bisuo a, Blephamide, Bronal, Capsoid, Cetapred, Chloramphecort-h, Compesolon, Cor tyzine, Corotrope, Cortan, Cortico-sol, Cortisal, Cortisol, Danalone, Decortin h, Delta-cortef, Deltacortenesol, Deltacortril, Deltahydrocortisone, Deltapred, Deltastab, Dermol, Dermosolon, Deturgylone, Dhasolone, Di-adreson-f, Dojilon, Dontisolon, Econopred, Emsolone, Encortolon, Estilsona, Fenicort, Fisiopred, Fisopred, Flo-pred, Frisolona forte, Glucortin, Gupisone, Hefasolon, Hexacorton, ...
Prednisolone (Solu-decortin). Prednisolone is used for treating allergies, arthritis, breathing problems (eg, asthma), certain blood disorders, collagen diseases (eg, lupus), certain eye diseases (eg, keratitis), cancer (eg, leukemia), endocrine problems (eg, adrenocortical insufficiency), intestinal problems (eg, ulcerative colitis), swelling due to certain conditions, or skin conditions (eg, psoriasis).. Other names for this medication. Adelcort, Adelone, Aersolin d, Ak-pred, Alertine, Alpicort, Apicort, Aprednislon, Bisuo a, Blephamide, Bronal, Capsoid, Cetapred, Chloramphecort-h, Compesolon, Cor tyzine, Corotrope, Cortan, Cortico-sol, Cortisal, Cortisol, Danalone, Decortin h, Delta-cortef, Deltacortenesol, Deltacortril, Deltahydrocortisone, Deltapred, Deltastab, Dermol, Dermosolon, Deturgylone, Dhasolone, Di-adreson-f, Dojilon, Dontisolon, Econopred, Emsolone, Encortolon, Estilsona, Fenicort, Fisiopred, Fisopred, Flo-pred, Frisolona forte, Glucortin, Gupisone, Hefasolon, Hexacorton, ...
Prednisolone is used for treating allergies, arthritis, breathing problems (e.g., asthma), certain blood disorders, collagen diseases (e.g., lupus), certain eye diseases (e.g., keratitis), cancer (e.g., leukemia), endocrine problems (e.g., adrenocortical insufficiency), intestinal problems (e.g., ulcerative colitis), swelling due to certain conditions, or skin conditions (e.g., psoriasis ...
Alarsin R.Compound is indicated in Rheumatic, Arthritic, Collagen diseases. Acute Soft Tissue Trauma. Dental Traumatic conditions. - Arthritis / Joint / Knee
Prednisolone is used for treating allergies, arthritis, breathing problems (e.g., asthma), certain blood disorders, collagen diseases (e.g., lupus), certain eye diseases (e.g., keratitis), cancer (e.g., leukemia), endocrine problems (e.g., adrenocort ...
Alport syndrome (AS) is a hereditary type IV collagen disease that leads to progressive proteinuria, renal fibrosis, and kidney failure. Depending on the mutated gene and the pattern of inheritance, there are three types ...
Hydrocortisone (Science: chemical) (11 beta)-11,17,21-trihydroxypregn-4-ene-3,20-dione. The main glucocorticoid secreted by the adrenal cortex. Its synthetic counterpart is used, either as an injection or topically, in the treatment of inflammation, allergy, collagen diseases, asthma, adrenocortical deficiency, shock, and some neoplastic conditions. Pharmacological action: steroidal anti-inflammatory agents. chemical name: Pregn-4-ene-3,20-dione, 11,17,21-trihydroxy-, (11beta)- ...
Identification of the molecular defect in patients with type II collagen disorders is usually a challenge because of the relatively large size and complexity of the COL2A1 gene and its main expression in cartilage. Cartilage is not easily accessible, relatively acellular, and therefore a poor source of mRNA for cDNA synthesis and analysis. Furthermore, sufficient amounts of mRNA can often not be obtained from cultured skin fibroblasts or lymphocytes owing to low basal transcription of the gene. Therefore, mutation analysis of the COL2A1 gene is often performed in the genomic DNA in a "head to tail" fashion, which is time consuming and laborious. In this study, we used the opportunity of having cartilage tissue for exploring the genetic defect in the COL2A1 gene in seven patients with a lethal type II collagen disorder. Those regions of theCOL2A1 gene in which a mutation was likely to reside, based on the overmodification pattern of the type II collagen CB peptides, were screened first with SSCP ...
collagen refers to a group of proteins which are the main component of connective tissue and the most abundant protein in mammals making up 25-35% of total body protein ...
Clinical. Scleroderma is a collagen-vascular disease characterized by diffuse fibrosis, degenerative changes, and vascular abnormalities in the skin, articular structures and other organs (kidneys, lung, heart, gastrointestinal and skeletal muscles). Esophageal involvement occurs in 75 to 87% of patients with scleroderma. In the esophagus, aperistalsis results from atrophy or cellular disruption of esophageal smooth muscle including the LES. These changes are primarily limited to the lower two-thirds of the esophagus because the upper third is composed of striated muscle. Patients often present with symptoms of reflux secondary to LES incompetence and dysphagia may result from abnormal motility, reflux esophagitis or peptic stricture formation. Radiological findings. Barium studies may demonstrate a normal stripping wave that clears the upper esophagus, but stops at the level of the aortic arch. This is can be attributed to the striated muscle that composes the upper third of the esophagus. In ...
Neurologic symptoms similar with those in multiple sclerosis appear in collagen-vascular diseases, Lyme disease, rare hereditary disorders, and AIDS so, although blood tests are highly recommended th...
All patients in the ONTT had blood testing to exclude collagen vascular disease (antinuclear antibody), syphilis (FTA-ABS), and a chest x-ray to detect sarcoidosis. A lumbar puncture was optional and was performed in 141 patients. Antinuclear antibody was positive in a titer 1:320 or greater in 3% of patients. Of these patients, only one developed a connective tissue disease in the first two years of follow up. FTA-ABS was positive in 1.3% of the patients, and none were judged to have syphilis. A chest x-ray did not SHOW evidence of sarcoidosis in any patients. CSF analysis never yielded unsuspected information. Based on these results, chest x-ray, blood tests, and lumbar puncture are deemed not to be necessary in evaluating patients with typical clinical features of optic neuritis ...
Cavitary pneumonia is a disease in which the normal lung architecture is replaced by a cavity. In a healthy lung, oxygen transport occurs at the level of the alveoli, each of which has an average size of 0.1 mm. These air spaces can become enlarged by a number of processes: bacterial infection (tuberculosis), fungal infection, vasculitis (granulomatosis with polyangiitis), collagen vascular disease (Sjögrens syndrome) or granulomatous disease (sarcoidosis). ...
Evidence of areas of irregular predominately peripheral consolidation in the right lower lobe with few areas of ground glass haze and nodular shadows. There is larger shadow in the left lower lobe with peripheral consolidation with airbronchogram surrounding relatively preserved lung parenchyma. There are areas of ground glass haze. The appearance in left lower lobe is suggestive of reverse halo sign or atoll sign which is classical of cryptogenic organising pneumonia versus differentials of this appearance. The cryptogenic organizing pneumonia in the majority of cases is an idiopathic disease, while less frequently it can be associated with infectious pneumonias, airway obstruction, aspiration, drug reaction, or collagen vascular disease associated with an intake of toxic substances. The prognosis is favourable because there is a good response to steroid therapyThis needs clinical correlation and follow up. ...
Author:Parul Vadgama, Mehul Bhavsar, Kusum V Shah, Rajesh Solanki, Ramakant Dixit, Jignesh Vaishnani. Keywords:Collagen vascular disease, pulmonary symptoms, sceroderma, rheumatoid arthritis. Type:Original Article. Full PDF Abstract ...
Gammadyn Cu-Au-Ag can be used to help arthritis, chronic rheumatism, skin and collagen disorders such as psoriasis and acne, immune depletion and infectious diseases
Vascular disorders can be classified into five groups: traumatic, compressive, occlusive, tumors/malformations and vasospastic. These problems may occur more commonly in individuals with certain diseases such as diabetes, hypertension, or kidney failure, or in dialysis patients.
This volume on CVDs, renal, and respiratory disorders has particularly high value. It carries the potential to become the most effective game-changer in global health by helping all countries to combat, contain, and control the biggest killer presently prowling the globe and by enabling us to reach the 2030 goals for NCDs and health overall. As one who has witnessed the epidemic of CVDs advance menacingly across the world in the past four decades, I fervently hope that the clear and convincing messages conveyed by the extensively researched and elegantly communicated analyses in this volume will be heard, heeded, and harmonized with policy and practice in all countries ...
This is the first prospective study comparing the diagnostic accuracy of cineradiography and infused intraluminal manometry in approximately equal numbers of patients with scleroderma, polymyositis, systemic lupus erythematosus (SLE), and rheumatoid arthritis. Major criteria for inclusion of patients were as follows: scleroderma, characteristic generalized skin and visceral lesions; polymyositis, proximal muscle weakness and characteristic biopsy, rash, or electromyogram; SLE, positive antinuclear factor with several of the following-polyarthritis, butterfly rash, unexplained nephritis, or polyserositis; and rheumatoid arthritis, classical disease as diagnosed by American Rheumatism Association criteria. No patients had dysphagia at the time of evaluation. After classification, esophageal manometry and cineradiography ...
A 66-year-old male complained of thickening of the skin at the face, posterior neck and back after a febrile episode. The patient had obesity, elevated levels of HbA1C and urinary C-peptide. The seological tests for collagen diseases were negative. The serum level of vascular endothelial growth factor (VEGF) was elevated. The biopsied skin specimen revealed the thickening of the dermis by the increased proliferation of collagen fibers, thickening of collagen bundles with fenestrations and infiltration of lymphocytes. Scleredema caused by diabestes mellitus and obesity was diagnosed, accompanied with insulin resistance. The patient was initially treated with administration of prednisolone, followed with diet therapy. Scleredema ameliorated partially, however elevated level of VEGF persisted after 6 months of discharge. It is unclear whether elevated level of VEGF may be related to the pathogenesis of the disease or may be an aggravating factor ...
Connective tissue diseases by Graham R. V. Hughes; 4 editions; First published in 1977; Subjects: Collagen diseases, Connective Tissue Diseases, Connective Tissue, Diseases, Connective tissues, Internet Archive Wishlist
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ogren syndrome, Sjogren-Larsson syndrome, Skeletal dysplasia, Skeletal muscle disease, Skin collagen disease, Skin vascular disease, Sleep disorder, Sleeping seekness, Sly disease, Small bowel adenocarcinoma, Small bowel leiomyosarcoma, Small non-cleaved cell lymphoma, Smith martin dodd syndrome, Smith-Fineman-Myers syndrome, Smith-Lemli-Opitz syndrome, Smith-Magenis syndrome, Sneddon syndrome, Sneddon-Wilkinson disease, Snyder-Robinson syndrome, Soft tissue perineurioma, Soft tissue sarcomas, Sohval soffer syndrome, Solitary plasmacytoma, Solomon syndrome, Somatotroph adenoma, Sommer hines syndrome, Sommer rathbun battles syndrome, Sommer-Young-Wee-Frye syndrome, Sondheimer syndrome, Sonoda syndrome, Sorsby syndrome, Sorsbys fundus dystrophy, Sotos syndrome, Spastic paraplegia, Spellacy gibbs watts syndrome, Spherophakia-brachymorphia, Sphingolipidosis, Spina bifida, Spinal atrophy, Spirillosis, Splenic marginal zone lymphoma, Spondylarthropathy, Spondylo camptodactyly syndrome, Spondylocostal ...
Collagen diseases such as systemic lupus erythematosis, Sjogrens syndrome, scleroderma and arthritis also cause muscular pain in the head and neck. Lupus is characterized by a butterfly rash of the face, fever, rheumatoid arthitis and pleural and abdominal pain. Laboratory studies reveal a high sedimentation rate, hypochromic anemia, a positive ANA test and a false-positive VDRL. Scleroderma is characterized by gradual onset of muscle and joint pain leading to a systemic sclerosis with anorexia, dyspnea and diminished sweating. Fever, skin lesions and limited jaw and chest expansion are seen. Sjogrens syndrome is characterized by dryness of the mouth, eye and skin and oftentimes, muscle and joint pain. Rheumatoid arthritis is a chronic inflammation of the synovial membrane with pain in the muscles and joints. Numerous joints in the body are usually affected, and pain usually decreases with use. Fatigue, fever, muscle pain, night sweats and sometimes weight loss are seen. Studies show the ...
In 1875 Paget described subclavian vein thrombosis and in 1884 Schröetter proposed that subclavian vein thrombosis was the result of excessive upper extremity activity. In 1949, the surgeon Hughes coined the term "Paget-Schröetter syndrome" for patients with occlusion of the subclavian vein following overuse of the upper extremity [1, 3, 6].. DVT occurs more commonly in the veins of the pelvis or the lower extremities. In two previous reports, the incidence of upper extremity DVT has been cited as occurring in approximately 4 to 11 % of all cases of thromboembolism. DVT is classified as either primary or secondary, depending of the cause of the thrombus. Primary or idiopathic upper extremity DVT is less common than secondary, only accounting for 2 in 100,000 cases per year [7, 8]. Secondary DVT is more common and can be caused by central venous catheters, cardiac devices, neoplasias, or collagen diseases. Thrombosis of the upper extremity veins is usually related to effort in conjunction with ...
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Patients with existing blood vessel aneurysms (such as abnormalities in brain blood vessels or aortic aneurysms), atherosclerotic disease ("hardening" of the arteries, including carotid artery disease), collagen disorders, vasculitis, other underlying blood vessel abnormalities, or collagen vascular diseases (such as systemic lupus erythematosus) may be at increased risk of stroke or blood vessel dissection (525;526). Individuals with osteomyelitis (bone infection) (527), cancer involving bone (502), vertebral fractures, severe degenerative joint disease (osteoarthritis), osteoporosis, and ankylosing spondylitis may be at increased risk of fracture or spinal damage leading to nerve disorders or spinal cord damage (528-530). Prior surgery of the trachea or tracheostomy may increase the risk of tracheal rupture (531). Underlying tumors of the brain or near the spinal cord may result in adverse outcomes such as tumor rupture or delayed diagnosis (532-537). Patients with blood clotting disorders or ...
A total of 187 specimens from 142 subjects with rheumatoid arthritis, systemic lupus erythematosus, other connective tissue diseases, and controls were placed in cell culture. Specimens from 119 of the subjects grew, lasting over 2 yr in several instances. No evidence of virus infection has been found by a variety of sensitive methods, including cell fusion. Other approaches have likewise failed thus far to implicate any virus in the pathogenesis of rheumatoid arthritis.. ...
ICD M30-M36 Systemic connective tissue disorders  autoimmune disease NOS collagen (vascular) disease NOS systemic autoimmune disease systemic collagen
BIOWARFARE RESEARCH Between 1942 and the present time, biological warfare research has resulted in a more deadly and infectious form of the mycoplasma. They extracted this mycoplasma from the brucellosis bacteria, weaponized it and actually reduced the disease to a crystalline form. According to Dr. Shyh-Ching Lo, one of Americas top, top researchers, this disease agent, the mycoplasma, causes among other things, AIDS, chronic fatigue syndrome, multiple sclerosis, Wegeners disease, Parkinsons disease, Crohns colitis, Type I diabetes, and collagen-vascular diseases such as rheumatoid arthritis and Alzheimers. The mycoplasma enters into the individual cells of the body depending upon your genetic predisposition. You may develop neurological diseases if the pathogen destroys certain cells in your brain, or you may develop Crohns colitis if the pathogen invades and destroys cells in the lower bowel. Once it gets into the cell, it can lie there doing nothing sometimes for 10, 20 or 30 years, ...
The Department of Rheumatology at Columbia Asia Hospital - Whitefield, Bangalore provides diagnosis, treatment and rehabilitation for adults and children with rheumatic and related disorders. Chronic diseases like rheumatoid arthritis, osteoarthritis, gout, pseudogout and fibromyalgia are some of the diseases commonly treated. Other diseases managed are collagen vascular diseases like Systemic Lupus Erythematosus (SLE), Poly Arteritis Nodosa (PAN) and Sjögrens syndrome. Rheumatic diseases affect many parts of the body, hence multi-disciplinary care is required and treatment is customized for each patient. The consultants also use different modes of therapy like pain medications, anti-inflammatory medications, chemotherapeutic agents and intra-articular steroids.. The focus is on alleviating pain, improving quality of life and prolonging the life expectancy of patients with chronic rheumatic diseases. The department is supported by physiotherapists and occupational therapists.. ...
Have compromised ability for wound healing, such as: malnutrition, oral steroid use, history of collagen vascular disease (e.g. lupus, scleroderma, history of keloid formation (raised and thickened scars), atrophic dermatitis (extreme skin irritation or itchiness) or immunologic abnormalities such as vitiligo (white patches of skin due to loss of pigment ...
Introduction. Recent advances in therapy make early identification of patients with Pulmonary Hypertension (PH) important. The 2008 consensus statement reported an average UK treatment rate of 24.9 per million.(1). Aim. Our institution is a District General Hospital serving a population of 330,000. Our aim was to compare our treatment rate to the national average since the appointment of a physician with an interest in PH.. Method. Retrospective casenote review of patients attending our monthly PH clinic since 2006.. Results. We identified 102 Outpatient and 35 Inpatient referrals. 28 went on to receive specialist treatment for PH; mean age 66 (range 36-85); 24 female 4 male. Diagnoses: Chronic thromboembolic 10, Idiopathic 8, Collagen vascular disease 5, Congenital left to right shunt 2, COPD 2 and Portal hypertension 1. Haemodynamics (mean): Cardiac catheter: mean Pulmonary Artery Pressure (PAP) 48mmHg (n=22, range 22-78), cardiac output 4.8 (n=21, range 2.5-7.7), cardiac index 2.83 (n=22, ...
In an effort to understand the reasons behind people of any age, including children, being struck down with Rheumatoid Arthritis (RA) and other connective tissue diseases, we have delved and found some valuable and exciting conventional medical research.. The book The New Arthritis Breakthrough chronicles some 50 years of research and successful treatment of over 10,000 patients by the eminent US doctor Thomas McPherson Brown. It cites clinical trials that have repeatedly shown the link between bacteria and the inflammatory process that causes RA and other connective tissue diseases. Trial results show how these debilitating conditions are responsive to low dose antibiotic therapy. Patient reports in the book tell how antibiotic therapy has allowed them to discontinue cortisone, anti inflammatories and immuno-suppressant medications and regain a normal life after years of pain, debility, surgery, unemployment and untold difficulty.. In addition to results that are thought impossible for ...
You need to find a doctor who will listen...good luck finding one who keeps their ego in check. University Hospital ER rooms are often good places because I have found that they are so overly careful. OU and OSU in Oklahoma have great specialist. I have all the same symptoms and went years being told I had various weird but normal problems. After an ER visit because I felt I was dying, the Doc kept me for several days. During this time he discovered a 38 year old secret...I had lived with Ehlers Danlos my entire life without a diagnosis. There are six types, but in a nutshell, they are all Collagen disorders. This means the body doesnt make collagen properly. This causes tons of problems as 80 percent of the body is collagen. It is very painful, but we look normal, not only normal, but good! But we hurt, those nodes, those are common with Ehlers, we live with these problems, have surgery when we need it, but only when we need it! Look it up. If Im wrong what will it hurt? People are ignoring ...
Article from Fibromyalgia News Today (FNT) - Skin of Fibromyalgia Patients Could Provide Disease Clue Id like to share this interesting, but short article that has expanded my opinion about the link between Fibro, inflammatory/autoimmune disorders and Connective Tissue/Collagen Disorders. Via this link: Skin of Fibromyalgia Patients Could Provide Disease Clues Via: Fibromyalgia News Today.…
In Taiwan, ,2,000,000 people have a habit of chewing betel quids and an increasing number are adolescents (32) . This habit has resulted in an increase in the incidence of oral carcinoma (33) and OSF (34) . Although the etiology of OSF remains unclear, evidence has shown that it is a collagen disorder related to betel quid exposure (14, 15, 16, 17 , 22) . The obvious bimodal distribution in the betel quid exposure among OSF patients, as shown in Fig. 1⇓ , suggests individual differences in susceptibility to the development of OSF. It is essential to evaluate the OSF risk associated with polymorphisms of collagen-related genes in low- and high-exposure groups separately to examine whether the high-risk genotypes are consistent or not. Because several genes are involved in the metabolism and cross-linking of collagens, it is, therefore, important to evaluate the combined effects of multiple genes as well as the main effect of single genes. To the best of our knowledge, this is the first study to ...