We read with great interest the article by Tack et al on the effect of the selective serotonin reuptake inhibitor (SSRI) citalopram on symptoms in patients with irritable bowel syndrome (IBS) (Gut 2006;55:1095-103). The usefulness of the results of this study are however debatable. Several previous studies have investigated the effect of tricyclic antidepressants and SSRIs on functional gastrointestinal symptoms. Because of errors or lack of clarity in study design, inclusion of very selected patient populations and, above all, small sample sizes, their role in the treatment of patients with IBS in daily clinical practice remains unclear.. The study of Tack et al, as already correctly pointed out by Creed in his commentary (Gut 2006;55:1065-7), also suffers from major shortcomings in study design, poor description of study population and no information on whether or not subjects and physicians/investigators were blinded and, if yes, how ...
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The researchers also measured depression and anxiety scores.. The effect of acute administration of citalopram on colonic sensitivity and on colonic response to feeding was assessed as a predictor of response to the drug.. The research team found after 3 and 6 weeks of treatment that citalopram improved abdominal pain, and bloating.. Impact of symptoms on daily life, and overall well being compared was also improved after 3 and 6 weeks with citalopram.. There was only a modest effect on stool pattern.. Changes in depression or anxiety scores were not related to symptom improvement.. The team noted that the effect of acute administration of citalopram during a colonic barostat study did not predict clinical outcome.. The researchers confirmed the benefit of citalopram over placebo after the analysis of the first treatment period as a double blind parallel arm study.. Dr Tacks team concluded, The selective serotonin reuptake inhibitors citalopram significantly improves irritable bowel syndrome ...
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Serotonin has been suggested to be important in human development, especially cardiac and craniofacial developments [18,19,20]. Despite the safety concerns about the effects of SSRIs on human development in utero, in general, these drugs are preferred over tricyclic antidepressants for the treatment of depression during pregnancy because they are known to be safer and have fewer adverse effects. Among SSRIs, citalopram is especially known to have few interactions in concomitant use with other drugs and is selects for 5-hydroxytryptamine uptake inhibition [21,22]. The trough plasma concentration of citalopram is even lower in pregnant women compared to non-pregnant women. This is likely a result from volume expansion and distribution and reduced plasma protein binding during pregnancy [23]. The lower plasma concentration of citalopram during pregnancy and its selectivity imply a theoretically lower teratogenicity of this drug; however, some controversy about the association of citalopram with ...
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600 stroke patients will be randomised to either escitalopram or placebo treatment in a 1:1 ratio and genotyped according to SERT polymorphisms. The treatment and follow up period is 6 months. During these 6 months there will be 2 clinical follow up visits, one telephone control and one visit to evaluate compliance regarding medication. Patients who had an MRI as a part of the routine investigations done upon admission (approximately 300 patients) will have a control MRI after 6 months.. Additionally 400 patients, not eligible for participation i the randomised controlled trial, will be genotyped and answer questionnaires after 1 and 6 months.. ...
... ! Citalopram is used to treat depression. Citalopram is in a class of antidepressants called selective serotonin reuptake inhibitors (SSRIs). It is thought to work by increasing the amount of serotonin, a natural substance in the brain that helps maintain mental balance.
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Serotonin signaling suppresses generation of amyloid-β (Aβ) in vitro and in animal models of Alzheimers disease (AD). We show that in an aged transgenic AD mouse model (APP/PS1 plaque-bearing mice), the antidepressant citalopram, a selective serotonin reuptake inhibitor, decreased Aβ in brain interstitial fluid in a dose-dependent manner. Growth of individual amyloid plaques was assessed in plaque-bearing mice that were chronically administered citalopram. Citalopram arrested the growth of preexisting plaques and reduced the appearance of new plaques by 78%. In healthy human volunteers, citaloprams effects on Aβ production and Aβ concentrations in cerebrospinal fluid (CSF) were measured prospectively using stable isotope labeling kinetics, with CSF sampling during acute dosing of citalopram. Aβ production in CSF was slowed by 37% in the citalopram group compared to placebo. This change was associated with a 38% decrease in total CSF Aβ concentrations in the drug-treated group. The ...
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Escitalopram, like other SSRIs, has been shown to affect sexual functions causing side effects such as decreased libido, delayed ejaculation, genital anesthesia,[17] and anorgasmia.[18][19]. An analysis conducted by the FDA found a statistically insignificant 1.5 to 2.4-fold (depending on the statistical technique used) increase of suicidality among the adults treated with escitalopram for psychiatric indications.[20][21][22] The authors of a related study note the general problem with statistical approaches: due to the rarity of suicidal events in clinical trials, it is hard to draw firm conclusions with a sample smaller than two million patients.[23]. Escitalopram is not associated with significant weight gain. For example, 0.6 kg mean weight change after 6 months of treatment with escitalopram for depression was insignificant and similar to that with placebo (0.2 kg).[24] 1.4-1.8 kg mean weight gain was reported in 8-month trials of escitalopram for depression,[25] and generalized anxiety ...
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NG Citalopram: Citalopram belongs to a group of medications called selective serotonin reuptake inhibitors (SSRIs). It is used to treat depression. SSRIs improve depression by increasing the amount of serotonin (a neurotransmitter) in certain areas of the brain, which in turn improves the ability of the brain to transmit messages from one nerve cell to another.
CO Citalopram: Citalopram belongs to a group of medications called selective serotonin reuptake inhibitors (SSRIs). It is used to treat depression. SSRIs improve depression by increasing the amount of serotonin (a neurotransmitter) in certain areas of the brain, which in turn improves the ability of the brain to transmit messages from one nerve cell to another.
... ! Citalopram belongs to a group of medications called selective serotonin reuptake inhibitors (SSRIs). Increased serotonin levels can help improve mood.
Citalopram is a selective serotonin reuptake inhibitor, or SSRI, that is commonly prescribed for the treatment of depression, according to WebMD. Citalopram is the generic version of Celexa and is...
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Objective:. Major depressive disorder (MDD) is a serious, debilitating, life-shortening illness that affects many persons of all ages and backgrounds. Although many patients suffering from MDD can expect improvement with antidepressant treatment, only a minority experience full remission, and little is known about the basis for individual differences in treatment outcome. The rationale for this study follows from the findings that treatment response in major depression is associated with: 1) variation in the genes encoding the serotonin 2A receptor (HTR2A) and the KA1 subunit of the glutamate-kainate receptor (GRIK4); and 2) increases in prefrontal glutamate/glutamine (glx) concentration measured by [+H] magnetic resonance spectroscopy (MRS). The central hypothesis is that genes associated with citalopram treatment outcome modulate glutamate concentrations in prefrontal brain (PFB) and thus facilitate response to citalopram treatment. Such information is important because it would ultimately ...
Early life stress is a potential precursor of eventual neuropsychiatric diseases and may result in altered neurodevelopment and function of the hippocampus, which thus provides a site at which potential interventions to modify the effects of early life stress may act. In this study, Sprague Dawley rat pups comprising male and female animals underwent maternal separation (MS) for 180 min from postnatal days (PND) 2 to 14, or were left with their dams. They subsequently received daily administration of saline (0.9%), escitalopram (10 mg/kg), or no treatment during adolescence (PND 43-60). All adult animals underwent brain magnetic resonance imaging (MRI) and bilateral hippocampal proton magnetic resonance spectroscopy ((1)H-MRS). Neither MS nor escitalopram treatment had a significant effect on hippocampal volume. Adult rats that experienced MS displayed significantly increased choline-containing compounds (Cho) and decreased N-acetylaspartate (NAA), glutamate (Glu) and Myo-inositol (MI) relative ...
Escitalopram is the first antidepressant introduced according to chirality rules, it is an S-enantiomer of citalopram, the drug which has been used for many years. Experimental studies showed that the property of serotonin transporter inhibition - one of the main mechanisms of antidepressive action is connected with the S-enantiomer of citalopram, and that escitalopram is the most selective inhibitor of this transporter. The results of most clinical studies in patients with depression show significant superiority of escitalopram, 10-20 mg/day, over placebo, as early as within the first week of treatment, and a faster onset of action and higher therapeutic efficacy of escitalopram, compared to citalopram, 20-40 mg/day. A similar efficacy of escitalopram, 10mg/day, and sertraline 50-200 mg/day, as well as escitalopram, 20 mg/day, and venlafaxine, 225 mg/day was demonstrated. It has also been shown that escitalopram, 10-20 mg/day, exerts therapeutic efficacy in general anxiety disorder, panic ...
Background: Subsyndromal symptoms of depression (SSD) in older outpatients with schizophrenia are common and clinically important. While many physicians prescribe antidepressants to patients with schizophrenia and schizoaffective disorder who have SSD, evidence for their effectiveness and safety has been meager. We describe a randomized placebo-controlled trial of citalopram in 198 patients.. Method: Participants in this 2-site study, conducted from September 1, 2001, to August 31, 2007, were men and women with DSM-IV schizophrenia or schizoaffective disorder who were 40 years of age or older and who met study criteria for SSD. Patients were randomly assigned to flexible-dose treatment with citalopram or placebo augmentation of their current antipsychotic medication. Analysis of covariance was used to compare improvement in scores on the Hamilton Rating Scale for Depression and Calgary Depression Rating Scale between treatment groups; secondary efficacy analyses compared improvement in several ...
subpages}} {{TOC,right}} In [[psychiatry]], citalopram is a [[second-generation antidepressant]] for treating [[major depressive disorder]]. ==History== ==Efficacy== ==Pharmacology== ===Administration=== ===Distribution=== ===Metabolism=== Citalopram is metabolised by [[cytochrome P-450]] CYP3A4 and [[Cytochrome P-450 CYP2C19,CYP2C19]]. ===Excretion=== ==Toxicity== [[Drug toxicity]] includes arrhythmias. The [[United States]] [[Food and Drug Administration]] has issued a warning to not use higher doses because of use of the [[cytochrome P-450]] system and also arrhythmias.,ref>{{cite web ,url= http://www.fda.gov/Safety/MedWatch/SafetyInformation/SafetyAlertsforHumanMedicalProducts/ucm297624.htm ,title=Safety Alerts for Human Medical Products > Celexa (citalopram hydrobromide) - Drug Safety Communication: Revised Recommendations, Potential Risk of Abnormal Heart Rhythms ,author=anonymous ,work=fda.gov ,year=2012 [last update] ,accessdate=March 29, 2012}},/ref> ==External links== ...
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