Abstract A congenic rat strain (WF.LEW) was derived from the susceptible Wistar-Furth (WF) (background strain) and the resistant LEW (donor strain) inbred strains and was used to evaluate the phenotypic expression of a dominant Mendelian gene that confers resistance to fatal hepatic disease caused by the ZH501 strain of Rift Valley fever virus (RVFV). Resistance to hepatic disease developed gradually with age, with full expression at approximately 10 weeks in the WF.LEW and LEW rat strains. The ZH501 strain caused fatal hepatitis in WF rats regardless of age. However, resistance to the SA75 RVFV strain (relatively non-pathogenic for adult rats), was age- and dose-dependent in both WF and LEW rats. The resistance gene transferred to the newly derived WF.LEW congenic rat strain appears to amplify age-dependent resistance of adult rats, resulting in protection against fatal hepatic disease caused by the virulent ZH501 strain. The congenic rat strain will be a valuable asset in elucidating the mechanism of
Rat Mcs3 was physically confirmed and delimited to a 27.8 Mb segment of rat chromosome 1. Rat Mcs3 is the last of the known Cop rat mammary carcinoma resistance-associated QTL (Mcs1-3) to be physically confirmed. In their linkage analysis predicting the Mcs3 QTL, Goulds group reported that Mcs3 heterozygous females had, on average, a 42% reduction in mammary carcinoma multiplicity compared to the WF phenotype, and females homozygous for the Mcs3 Cop allele had an 84% reduction in mammary tumor number (Shepel et al. 1998). Thus, they appropriately concluded that there was no dominance effect at the Mcs3 QTL. While we did not test heterozygous females in our study, we observed that the Mcs3 Cop allele reduced the mammary carcinoma susceptibility phenotype of the highly susceptible WF strain from 46 to 65% when homozygous. The discrepancy between homozygous genotypes in these two studies may be due to effects of Cop resistance alleles at other Mcs QTL present in the linkage analysis study, as the ...
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ORAL PRESENTATION THURSDAY OCTOBER 21 9.30am - 9.45am MECHANISM OF RESISTANCE TO PLASMODIUM CHABAUDI IN MICE IS MEDIATED THROUGH THE RED CELL AND A TOTALLY SYNERGISTIC NON-ERYTHROCYTIC PATHWAY Lin E, Marshall V, Burt RA, Foote SJ The Walter & Eliza Hall Institute, Parkville, Australia P. chabaudi infection in mice is a model for the severe anaemia seen in falciparum infections. Inbred mice strains are differentially susceptible to malaria. We have mapped three loci involved in outcome to this infection and have generated mice reciprocally congenic for these loci on several strains of mouse. These congenic animals have phenotypes different from their wildtype parents. Parasitaemias are more informative than clinical outcome in differentiating between the congenic animals. In one case, a congenic animal carrying a locus predicted to encode susceptibility was much more resistant than even the "resistant" animal. Analysis of the ability of red cells from congenic mice to sustain the growth of the ...
Autoimmune type 1 diabetes (T1D) in humans and NOD mice results from interactions between multiple susceptibility genes (termed Idd) located within and outside the MHC. Despite sharing ∼88% of their genome with NOD mice, including the H2(g7) MHC haplotype and other important Idd genes, the closely related nonobese resistant (NOR) strain fails to develop T1D because of resistance alleles in residual genomic regions derived from C57BLKS mice mapping to chromosomes (Chr.) 1, 2, and 4. We previously produced a NOD background strain with a greatly decreased incidence of T1D as the result of a NOR-derived 44.31-Mb congenic region on distal Chr. 4 containing disease-resistance alleles that decrease the pathogenic activity of autoreactive B and CD4 T cells. In this study, a series of subcongenic strains for the NOR-derived Chr. 4 region was used to significantly refine genetic loci regulating diabetogenic B and CD4 T cell activity. Analyses of these subcongenic strains revealed the presence of at least two
We have previously reported suggestive evidence for a locus on Chromosome (Chr) 7 that affects adiposity in F2 mice from a CAST/Ei x C57BL/6J intercross fed a high-fat diet. Here we characterize the effect of a high-fat (32.6 Kcal% fat) diet on male and female congenic mice with a C57BL/6J background and a CAST/Ei-derived segment on Chr 7. Adiposity index (AI) and weights of certain fat pads were approximately 50% lower in both male and female congenic mice than in control C57BL/6J mice, and carcass fat content was significantly reduced. The reduction of fat depot weights was not seen, however, in congenic animals fed a low-fat chow diet (12 Kcal% fat). The congenic segment is approximately 25 cM in length, extending from D7Mit213 to D7Mit41, and includes the tub, Ucp2 and Ucp3, genes, all of which are candidate genes for this effect. Some polymorphisms have been found on comparing c-DNA sequences of the Ucp2 gene from C57BL/6J and CAST/Ei mice. These results suggest that one or more genes
Congenic strains are produced by transferring the transgene/KO allele to a new genetic background strain that is more appropriate for phenotypic analysis. The APF offer a speed congenics service that can establish a congenic strain within 18 months.
BACKGROUND: Complex etiology and pathogenesis of pathophysiological components of the cardio-metabolic syndrome have been demonstrated in humans and animal models. METHODOLOGY/PRINCIPAL FINDINGS: We have generated extensive physiological, genetic and genome-wide gene expression profiles in a congenic strain of the spontaneously diabetic Goto-Kakizaki (GK) rat containing a large region (110 cM, 170 Mb) of rat chromosome 1 (RNO1), which covers diabetes and obesity quantitative trait loci (QTL), introgressed onto the genetic background of the normoglycaemic Brown Norway (BN) strain. This novel disease model, which by the length of the congenic region closely mirrors the situation of a chromosome substitution strain, exhibits a wide range of abnormalities directly relevant to components of the cardio-metabolic syndrome and diabetes complications, including hyperglycaemia, hyperinsulinaemia, enhanced insulin secretion both in vivo and in vitro, insulin resistance, hypertriglyceridemia and altered pancreatic
Complete information for BP16 gene (Genetic Locus), Blood Pressure QTL 16, including: function, proteins, disorders, pathways, orthologs, and expression. GeneCards - The Human Gene Compendium
Organisms that differ in genotype at (ideally) one specified locus. Strictly speaking these are conisogenics. Thus one homozygous strain can be spoken of as being congenic to another
UW-Madison. We have used both classical genetics and sequence-based genomics in search of mouse modifiers of liver tumorigenesis. The dramatic 20-50-fold difference in tumor multiplicity between carcinogen-treated male C57BL/6 (B6) and C3H/HeJ (C3H) mice has been shown to map mainly to distal chromosome 1. We have bred congenic animals carrying 70cM of chromosome 1 from C3H on an otherwise B6 genetic background. Relative to B6 animals, these B6.C31 mice developed up to 14-fold more liver tumors. Analysis of recombinant animals carrying smaller portions of the C3H congenic region suggests the presence of two modifiers, one of which has a 5-8-fold effect on tumor multiplicity and lies in a 7 Mb region on distal chromosome 1. Ras mutations are more prevalent in C3H tumors than in B6. By comparing ras mutations in tumors from the B6.C31and parental strains, we have shown that the C3H alleles on distal chromosome 1 are not sufficient to recapitulate the high ras mutant frequency of C3H tumors, ...
Jétudie les liens existant entre lévolution de la composition nucléotidique des génomes mammifères et leurs traits dhistoire de vie.. J. Romiguier, E. Figuet, N. Galtier, E.J.P. Douzery, B. Boussau, V. Ranwez, J.Y. Dutheil. 2012. Fast and robust characterization of time-heterogeneous sequence evolutionary processes using substitution mapping. Plos One (accepted). J. Romiguier, V. Ranwez, E. Douzery, N. Galtier. 2010. Contrasting GC-content dynamics across 33 mammalian genomes: influence of body mass and chromosome size. Genome Research 20:1001-1009.. J.Y. Dutheil, N. Galtier, J. Romiguier, E.J.P. Douzery, V. Ranwez, B. Boussau. 2012. Efficient selection of branch-specific models of sequence evolution. Molecular Biology and Evolution (accepted). ...
Hall MA, Norman PJ, Thiel B, Tiwari H, Peiffer A, Vaughan RW, Prescott S, Leppert M, Schork NJ, Lanchbury JS, Quantitative trait loci on chromosomes 1, 2, 3, 4, 8, 9, 11, 12, and 18 control variation in levels of T and B lymphocyte subpopulations. Am J Hum Genet70(5):1172-82 ...
Renin has long been suspected to reside in a compound BP QTL, with multiple normotensive and hypertensive alleles that can be derived from a single strain.2 Our data indicate that at least 2 loci around Renin modulate BP (Figure 1). In addition to the BN protective Renin region (46.1-47.2 Mb), we also identified a BN hypertensive region (47.2-49.0 Mb), which in lines 9 and 9C masked the protective allele(s) that were observed in line 9A (Figure 1). Similar to line 9, the S/renrr congenic unexpectedly failed to lower BP compared with SS.11 The SR had a region (47.9-48.1 Mb) that was significantly enriched for common BN and SR alleles (Figure 3), suggesting that this overlapping BN and SR region are potentially functionally relevant.. The BP-associated Renin allele that was first identified by Rapp et al4 in 1989 turned out to be part of a much larger BP QTL (chr13: 35-111 Mb)5 that encompasses multiple BP loci on chromosome 13 in the SS5 and other hypertensive strains (eg, SHR28 and LH29). Using ...
The availability of high-density genotype data for mice has made it possible to quantify the relationship between haplotype structure and differential gene expression. A relationship between SNP in the 1 kb upstream region and differential expression was only observable when using a stringent test of differential expression (absolute log2 fold change , 0.5; pplr , 0.005). This was interpreted as evidence that the variance of expression of cis regulated genes is much lower than that of trans regulated genes. Although the 1 kb upstream region may contain the highest density of regulatory elements it does not contain all of them, they can be spread throughout the gene and its 3 region as well as going tens to hundreds of kilobases upstream. Therefore we are likely to have underestimated the numbers of cis regulated genes using this strategy. However SNP in the upstream region will frequently be markers for larger haplotypes that extend into or through the whole gene region so these regions will ...
The cdc2+ gene function plays a central role in the control of the mitotic cell cycle of the fission yeast Schizosaccharomyces pombe. Recessive temperature-sensitive mutations in the cdc2 gene cause cell cycle arrest when shifted to the restrictive temperature, while a second class of mutations within the cdc2 gene causes a premature advancement into mitosis. Previously the cdc2+ gene has been cloned and has been shown to encode a 34 kDa phosphoprotein with in vitro protein kinase activity. Here we describe the cloning of 11 mutant alleles of the cdc2 gene using two simple methods, one of which is presented here for the first time. We have sequenced these alleles and find a variety of single amino acid substitutions mapping throughout the cdc2 protein. Analysis of these mutations has identified a number of regions within the cdc2 protein that are important for cdc2+ activity and regulation. These include regions which may be involved in the interaction of the cdc2+ gene product with the proteins ...
Mapping studies have identified many QTLs affecting BP in genetically hypertensive rats, and their isolation in congenic strains has been the main approach used for their further characterization (20). Apart from genetic analysis, congenic strains provide a powerful tool for identifying relevant intermediary phenotypes, since they only differ from control (parental) strains for a small fraction of the genome. Thus any differences seen are more likely to be involved in the physiological pathway(s) that regulates the trait of interest. In this study, we demonstrate for the first time that the rat chromosome 1 BP QTL region also influences pressure-natriuresis relationship, salt sensitivity, and most probably sympathetic activation following salt loading. These findings may be related and pertinent to the effect on BP.. Pressure-natriuresis was studied using the classic method designed by Roman and Cowley (23), which one allows to maintain, at a fixed level, most of the extrarenal factors that ...
TY - JOUR. T1 - The Cystatin S gene maps to rat Chromosome 3, to which D1mgh18 is re-assigned from Chromosome 1. AU - Duran Alonso, M. Beatriz. AU - Shiels, Paul. AU - McCallion, Andrew S. AU - Bennett, Neil K.. AU - Payne, Anthony P.. AU - Szpirer, Josiane. AU - Szpirer, Claude. AU - Brodie, Martin J.. AU - Davies, R. Wayne. AU - Sutcliffe, Roger G.. PY - 1997/12. Y1 - 1997/12. UR - http://www.scopus.com/inward/record.url?scp=0031543282&partnerID=8YFLogxK. UR - http://www.scopus.com/inward/citedby.url?scp=0031543282&partnerID=8YFLogxK. M3 - Article. C2 - 9383294. AN - SCOPUS:0031543282. VL - 8. SP - 946. EP - 947. JO - Mammalian Genome. JF - Mammalian Genome. SN - 0938-8990. IS - 12. ER - ...
At file:///home/inaam/w/lru_flush/ based on revid:[email protected] 3387 Inaam Rana 2010-12-14 non-functional change: refactored buf_LRU_free_block() to reduce the level of indentation modified: storage/innobase/buf/buf0lru.c === modified file storage/innobase/buf/buf0lru.c --- a/storage/innobase/buf/buf0lru.c revid:[email protected] +++ b/storage/innobase/buf/buf0lru.c revid:[email protected] @@ -1472,6 +1472,7 @@ buf_LRU_free_block( buf_page_t* b = NULL; buf_pool_t* buf_pool = buf_pool_from_bpage(bpage); enum buf_lru_free_block_status ret; + enum buf_page_state page_state; const ulint fold = buf_page_address_fold(bpage-,space, bpage-,offset); rw_lock_t* hash_lock = buf_page_hash_lock_get(buf_pool, fold); @@ -1576,172 +1577,178 @@ func_exit: #endif /* UNIV_SYNC_DEBUG */ ut_ad(buf_page_can_relocate(bpage)); - if (buf_LRU_block_remove_hashed_page(bpage, zip) - != BUF_BLOCK_ZIP_FREE) { + page_state = buf_LRU_block_remove_hashed_page(bpage, zip); - /* We have just freed a ...
A general experimental design that allows mapping of a quantitative trait locus (QTL) into a 1-cM interval is presented. The design consists of a series of strains, termed
At file:///home/marko/innobase/dev/mysql-5.1-innodb2/ based on revid:[email protected] 3458 Marko Mäkelä 2010-05-11 Do not demand that buf_page_t be fully initialized on 64-bit systems. There may be padding before buf_page_t::zip. (Bug #53307) modified: storage/innodb_plugin/buf/buf0lru.c storage/innodb_plugin/include/buf0buf.ic === modified file storage/innodb_plugin/buf/buf0lru.c --- a/storage/innodb_plugin/buf/buf0lru.c 2010-03-23 16:20:36 +0000 +++ b/storage/innodb_plugin/buf/buf0lru.c 2010-05-11 10:50:12 +0000 @@ -1393,7 +1393,12 @@ buf_LRU_free_block( ut_ad(buf_page_in_file(bpage)); ut_ad(bpage-,in_LRU_list); ut_ad(!bpage-,in_flush_list == !bpage-,oldest_modification); +#if UNIV_WORD_SIZE == 4 + /* On 32-bit systems, there is no padding in buf_page_t. On + other systems, Valgrind could complain about uninitialized pad + bytes. */ UNIV_MEM_ASSERT_RW(bpage, sizeof *bpage); +#endif if (!buf_page_can_relocate(bpage)) { @@ -1688,7 +1693,12 @@ buf_LRU_block_remove_hashed_page( ...
These data establish linkage of rat genotype to a form of environmental perturbation-infection-that is potentially important in the pathogenesis of autoimmunity. They confirm that Iddm4 is an exceptionally strong non-MHC determinant of susceptibility to autoimmune diabetes in the rat (6,9-11). In previous studies of Iddm4, diabetes was induced by chronic treatment with poly I:C plus Treg depletion. The present data now extend the role of Iddm4 in diabetes pathogenesis to virus-induced disease expression. They also illuminate the complexity of environmental interaction with genetic susceptibility. The diabetogenic potential of Iddm4 is readily discernable in congenic rats treated with poly I:C and Treg depletion but is far less apparent in animals treated with KRV plus poly I:C unless additional BBDR genes are present. We have discovered at least one of these genes, designated Iddm20, on chromosome 17.. The Iddm20 interval (online appendix Table 3) contains at least one gene of particular ...
I work with rat tumors and try to establish their karyotype. Is there anyone who knows wher I can get such paints?? Bernd K lsch Cell-biology 45147 Essen, Germany e-mail: ttu3a0 at aixrs1.hrz.uni-essen.de knowq s where I can ...
This congenic strain carries the |i|H2|sup|b|/sup||/i| haplotype and |i|Fv1|sup|b|/sup||/i| allele from C57BL/6 on the AKR genetic background. Cells of strains carrying |i|Fv1|sup|b|/sup||/i| are resistant to N-tropic and susceptible to B-tropic viruses. |i|Fv1|sup|b|/sup||/i| is carried as the wild-type allele in the BALB/c, A and C57BL/6|br/| strains.|br/|
Trp53 gene knockout mice. A neomycin selection cassette was inserted into the second exon of the Trp53 gene. Homozygous deficient mice can grow after birth, but often suffered from development of tumors in various region of the body. Several congenic strains were generated. C57BL/6 background (RBRC01361), ICR background (RBRC01348), C3H background (RBRC00107), DBA/2 background (RBRC01518), and MSM background (RBRC00815). In C57BL/6 background most of the female homozygotes die in utero (probably, also in MSM background ...
Trp53 gene knockout mice. A neomycin selection cassette was inserted into the second exon of the Trp53 gene. Homozygous deficient mice can grow after birth, but often suffered from development of tumors in various region of the body. Several congenic strains were generated. C57BL/6 background (RBRC01361), ICR background (RBRC01348), C3H background (RBRC00107), DBA/2 background (RBRC01518), and MSM background (RBRC00815). In C57BL/6 background most of the female homozygotes die in utero (probably, also in MSM background ...
Weil, M. M., Brown, B. W., and Serachitopol, D. M. Genotype Selection to Rapidly Breed Congenic Strains. Genetics 146: 1061-1069 (July,1997). Language: Fortran 95. ...
We have investigated transport in a cross-shaped two-dimensional electron gas with superconducting electrodes coupled to two opposite arms. Multiterminal resistances, measured as a function of the superconducting phase difference and the magnetic flux, are analyzed in terms of an extended Landauer-Buttiker transport formalism. We show that extended reciprocity relations hold. Correlations between transport coefficients are obtained from, e.g., (negative) three-terminal and nonlocal resistances. Energy spectroscopy reveals a reentrant behavior of the transport coefficients around the Thouless energy ...
Quantitative trait loci (QTLs) have been mapped to small intervals along the chromosomes of tomato (Lycopersicon esculentum), by a method we call substitution mapping. The size of the interval to which a QTL can be mapped is determined primarily by the number and spacing of previously mapped genetic markers in the region surrounding the QTL. We demonstrate the method using tomato genotypes carrying chromosomal segments from Lycopersicon chmielewskii, a wild relative of tomato with high soluble solids concentration but small fruit and low yield. Different L. chmielewskii chromosomal segments carrying a common restriction fragment length polymorphism were identified, and their regions of overlap determined using all available genetic markers. The effect of these chromosomal segments on soluble solids concentration, fruit mass, yield, and pH, was determined in the field. Many overlapping chromosomal segments had very different phenotypic effects, indicating QTLs affecting the phenotype(s) to lie in ...
article{418b5550-2321-4395-ab0c-946f90945ceb, abstract = {OBJECTIVE: To characterize the arthritis-modulating effects of 3 non-major histocompatibility complex (MHC) quantitative trait loci (QTLs) in rat experimental arthritis in the disease-resistant E3 strain, and to investigate the disease-modulating effects of the MHC region (RT1) in various genetic backgrounds. METHODS: A congenic fragment containing Ncf1 along with congenic fragments containing the strongest remaining loci, Pia5/Cia3 and Pia7/Cia13 on chromosome 4, were transferred from the arthritis-susceptible DA strain into the background of the completely resistant E3 strain. The arthritis-regulatory potential of the transferred alleles was evaluated by comparing the susceptibility to experimental arthritis in congenic rats with that in E3 rats. The RT1(u) haplotype from the E3 strain was transferred into the susceptible DA strain (RT1(av1)), and various F(1) and F(2) hybrids were generated to assess the effects of RT1 on arthritis ...
It is well-documented that obesity participated in the development of renal cell carcinoma (RCC). Leptin is closely associated with obesity.
1 ---------- 2 SEMAPHORES 3 ---------- 4 OS WAIT ARRAY INFO: reservation count 36255, signal count 12675 5 --Thread 10607472 has waited at buf/buf0rea.c line 420 for 0.00 seconds the semaphore: 6 Mutex at 0x358068 created file buf/buf0buf.c line 597, lock var 0 7 waiters flag 0 8 --Thread 3488624 has waited at buf/buf0buf.c line 1177 for 0.00 seconds the semaphore: 9 Mutex at 0x358068 created file buf/buf0buf.c line 597, lock var 0 10 waiters flag 0 11 --Thread 6896496 has waited at btr/btr0cur.c line 442 for 0.00 seconds the semaphore: 12 S-lock on RW-latch at 0x8800244 created in file buf/buf0buf.c line 547 13 a writer (thread id 14879600) has reserved it in mode exclusive 14 number of readers 0, waiters flag 1 15 Last time read locked in file btr/btr0cur.c line 442 16 Last time write locked in file buf/buf0buf.c line 1797 [...] 17 Mutex spin waits 0, rounds 452650, OS waits 22573 18 RW-shared spins 27550, OS waits 13682; RW-excl spins 0, OS waits 0 ...
Read "A congenic mouse and candidate gene at the Chromosome 13 locus regulating bone density, Mammalian Genome" on DeepDyve, the largest online rental service for scholarly research with thousands of academic publications available at your fingertips.
Read "Increased physical activity cosegregates with higher intake of carbohydrate and total calories in a subcongenic mouse strain, Mammalian Genome" on DeepDyve, the largest online rental service for scholarly research with thousands of academic publications available at your fingertips.
define FUSE_USE_VERSION 26 #include ,sys/fsuid.h, #include ,sys/types.h, #include ,pthread.h, #include ,signal.h, #include ,limits.h, #include ,string.h, #include ,stdarg.h, #include ,stdlib.h, #include ,unistd.h, #include ,errno.h, #include ,fcntl.h, #include ,stdio.h, #include ,fuse.h, #include ,pwd.h, #include ,tcl.h, /* * Default cache directory */ #ifndef APPFS_CACHEDIR #define APPFS_CACHEDIR /var/cache/appfs #endif /* Debugging macros */ #ifdef DEBUG int appfs_debug_fd = STDERR_FILENO; #define APPFS_DEBUG(x...) { \ char buf[8192]; \ int bufoff = 0; \ if (appfs_debug_fd == -1) { \ appfs_debug_fd = open(/tmp/appfsd.log, O_WRONLY , O_APPEND , O_CREAT, 0600); \ }; \ bufoff = snprintf(buf, sizeof(buf), [debug] [t=%llx] %s:%i:%s: , (unsigned long long) pthread_self(), __FILE__, __LINE__, __func__); \ if (bufoff , sizeof(buf)) { \ bufoff += snprintf(buf + bufoff, sizeof(buf) - bufoff, x); \ }; \ if (bufoff , sizeof(buf)) { \ bufoff += snprintf(buf + bufoff, sizeof(buf) - bufoff, \n);\ } \ ...
Signed-off-by: Janne Grunau ,janne-ffmpeg at jannau.net, --- libavcodec/dvbsubdec.c , 11 +++++++++-- 1 files changed, 9 insertions(+), 2 deletions(-) diff --git a/libavcodec/dvbsubdec.c b/libavcodec/dvbsubdec.c index 401144f..c06c017 100644 --- a/libavcodec/dvbsubdec.c +++ b/libavcodec/dvbsubdec.c @@ -1423,13 +1423,15 @@ static int dvbsub_decode(AVCodecContext *avctx, #endif - if (buf_size ,= 2 ,, *buf != 0x0f) + if (buf_size ,= 6 ,, *buf != 0x0f) { + av_dlog(avctx, incomplete or broken packet); return -1; + } p = buf; p_end = buf + buf_size; - while (p , p_end && *p == 0x0f) { + while (p_end - p , 6 && *p == 0x0f) { p += 1; segment_type = *p++; page_id = AV_RB16(p); @@ -1437,6 +1439,11 @@ static int dvbsub_decode(AVCodecContext *avctx, segment_length = AV_RB16(p); p += 2; + if (p_end - p , segment_length) { + av_dlog(avctx, incomplete or broken packet); + return -1; + } + if (page_id == ctx-,composition_id ,, page_id == ctx-,ancillary_id ,, ctx-,composition_id == -1 ,, ctx-,ancillary_id == ...
BNX2]: Fix bug in bnx2_nvram_write(). Length was not calculated correctly if the NVRAM offset is on a non- aligned offset. Signed-off-by: Michael Chan ,[EMAIL PROTECTED], Signed-off-by: David S. Miller ,[EMAIL PROTECTED], --- drivers/net/bnx2.c , 4 ++-- 1 files changed, 2 insertions(+), 2 deletions(-) diff --git a/drivers/net/bnx2.c b/drivers/net/bnx2.c index f296c37..4fa7cef 100644 --- a/drivers/net/bnx2.c +++ b/drivers/net/bnx2.c @@ -3096,7 +3096,7 @@ bnx2_nvram_write(struct bnx2 *bp, u32 offset, u8 *data_buf, if ((align_start = (offset32 & 3))) { offset32 &= ~3; - len32 += align_start; + len32 += (4 - align_start); if ((rc = bnx2_nvram_read(bp, offset32, start, 4))) return rc; } @@ -3114,7 +3114,7 @@ bnx2_nvram_write(struct bnx2 *bp, u32 offset, u8 *data_buf, if (align_start ,, align_end) { buf = kmalloc(len32, GFP_KERNEL); - if (buf == 0) + if (buf == NULL) return -ENOMEM; if (align_start) { memcpy(buf, start, 4); - To unsubscribe from this list: send the line unsubscribe git-commits-head ...
Berikut Gambar Rangkaian 8951 dengan Handpone HP Siemens c45 List Program Assembly 8051 sebagai berikut : buf_rx data 0ffh buf_no data 0afh ;0a0h buf_psn data 0a0h ;8ch buf_psn_txt data 94h ;5ah buf_no_pdu data 88h ;46h -20 +1 NO_DIAL DATA 73H ;74 ;32h -15 +2 no_sms data 62h -20 +3 ;text_pdu data 4bh ;no_sms data 47h…
00119 { 00120 FILE *f; 00121 char buf[ 1024 ]; 00122 int i; 00123 static regexp *re_macros = 0; 00124 00125 00126 #ifdef OPT_IMPROVED_PATIENCE_EXT 00127 static int count = 0; 00128 ++count; 00129 if ( ((count == 100) ,, !( count % 1000 )) && DEBUG_MAKE ) 00130 printf("...patience...\n"); 00131 #endif 00132 00133 /* the following regexp is used to detect cases where a */ 00134 /* file is included through a line line "#include MACRO" */ 00135 if ( re_macros == 0 ) 00136 { 00137 re_macros = regex_compile( 00138 "^[ ]*#[ ]*include[ ]*([A-Za-z][A-Za-z0-9_]*).*$" ); 00139 } 00140 00141 00142 if( !( f = fopen( file, "r" ) ) ) 00143 return l; 00144 00145 while( fgets( buf, sizeof( buf ), f ) ) 00146 { 00147 for( i = 0; i , rec; i++ ) 00148 if( regexec( re[i], buf ) && re[i]-,startp[1] ) 00149 { 00150 re[i]-,endp[1][0] = \0; 00151 00152 if( DEBUG_HEADER ) 00153 printf( "header found: %s\n", re[i]-,startp[1] ); 00154 00155 l = list_new( l, newstr( re[i]-,startp[1] ) ); 00156 } 00157 00158 /* special ...
Whoa. This warning dates back to 2004, when MAX_BUF_SIZE was a thing that buffers.c enforced. But we removed MAX_BUF_SIZE back in 2007 when we replaced the buffer implementation ...
Vladimir Panteleev ,git at vladimir.panteleev.md, writes: , @@ -255,25 +260,31 @@ static void test_GetSetEnvironmentVariableW(void) , ok(ret == TRUE, should not fail with empty value but GetLastError=%d\n, GetLastError()); , , lstrcpyW(buf, fooW); , + SetLastError(0); You should probably use 0xdeadbeef here (and also in the A test that this was copied from). -- Alexandre Julliard julliard at winehq.org ...
Members brought some of their toughest cases to Dr. Lew Schon at this dynamic live event. Join us to hear his advice and the discussions that ensued for the following cases ...
BioMed Research International is a peer-reviewed, Open Access journal that publishes original research articles, review articles, and clinical studies covering a wide range of subjects in life sciences and medicine. The journal is divided into 55 subject-specific sections.
Compared with hexaploid wheat, tetraploid durum is more susceptible to Fusarium crown rot (FCR) infection. The feasibility of enhancing FCR resistance in durum wheat by introgressing chromosome segmen
Cell hybrids between malignant mouse hepatoma cells and normal rat fibroblasts with approximately one set of chromosomes from each parent exhibited remarkable karyotypic stability. Most chromosomes of both parents were retained even after prolonged culture in vitro. Normally, such hybrids showed suppression of the transformed phenotype and formed no colonies in soft agar. However, two hybrids, BS140 and BS181, formed a few colonies in soft agar when many cells were seeded, and also occasional foci of cells were detected piling up in monolayer cell cultures. We isolated soft agar colonies (a-subclones) and sub-clones from foci (h-subclones) of both hybrids, and, as a control, subclones of cells from random areas without foci of one hybrid (BS181 p-subclones). When tested for soft agar growth, cells from the a- and h-subclones of both BS140 and BS181 formed colonies at frequencies comparable to the malignant mouse hepatoma parent, whereas the control cells of the BS181 p-subclones (like the normal ...
Buffalo 0 0 2 1--3 Dallas 1 0 1 0--2 ------------------------------- FIRST PERIOD -- Scoring: 1, Dallas, Hull 6 (power play) (Modano, Lehtinen), 10:17. Penalties: Zubov, Dal (roughing), 6:36; Satan, Buf (boarding), 8:18; Patrick, Buf ( double high sticking minor), 12:46; D Ward, Buf (interference), 19:11. SECOND PERIOD -- Scoring: None. Penalties: Varada, Buf (goalie interference), 4:53; Zhitnik, Buf (interference), 7:07; D Ward, Buf (roughing), 9:34; Ludwig, Dal (hooking), 12:21; Matvichuk, Dal (interference), 16:33. THIRD PERIOD -- Scoring: 2, Buffalo, Barnes 5 (Juneau, Smehlik), 8:33. 3, Buffalo, Primeau 3 (power play) (Zhitnik, Smehlik), 13:37. 4, Dallas, Lehtinen 8 (Modano, Zubov), 19:11. Penalties: Sydor, Dal (tripping), 12:10; Mckee, Buf (charging), 14:17. OVERTIME -- Scoring: 5, Buffalo, Woolley 4 (Brown), 15:30. Penalties: Zhitnik, Buf (hooking), 6:41; Sanderson, Buf (boarding), 9:06. Shots on goal: ------------------------------------ Buffalo 5 4 10 5--24 Dallas 11 12 7 7--37 ...
Methodologies for mapping of genes underlying quantitative traits have advanced considerably but have not been accompanied by a parallel development of new population structures. We present a novel population consisting of 50 introgression lines (ILs) originating from a cross between the green-fruited species Lycopersicon pennellii and the cultivated tomato (cv M82). Each of the lines contains a single homozygous restriction fragment length polymorphism-defined L. pennellii chromosome segment, and together the lines provide complete coverage of the genome and a set of lines nearly isogenic to M82. A field trial of the ILs and their hybrids revealed at least 23 quantitative trait loci (QTL) for total soluble solids content and 18 for fruit mass; these estimates are twice as high as previously reported estimates based on traditional mapping populations. For finer mapping of a QTL affecting fruit mass, the introgressed segment was recombined into smaller fragments that allowed the identification of ...
Results 3 days after pristane application the proportion of cells positive for cleaved caspase 3 was highly increased in draining inguinal lymph nodes of DA.1F rats indicating apoptosis. Interestingly, the apoptosis rate decreased to normal levels as seen as in naïve animals within 2 weeks but draining lymph nodes became extremely hypercellular. In peripheral blood both apoptosis and necrosis rates doubled during the first week after pristane injection. In the acute disease phase four times increased apoptosis and necrosis rates were observed which was in contrast to the draining lymph nodes which normalised during the disease course. The authors could not observe any phagoytosis deficiency in pristane-injected animals rather a strongly increased phagocytosis activity was seen in the acute disease phase. In line with this in vivo data, pristane induced pronounced apoptosis and secondary necrosis in a dose dependent manner when added to cultured rat splenocytes.. ...
Pattern recognition receptors (PRRs) are crucial for responses to infections and tissue damage; however, their role in autoimmunity is less clear. Herein we demonstrate that 2 C-type lectin receptors (CLRs) Mcl and Mincle play an important role in the pathogenesis of experimental autoimmune encephalomyelitis (EAE), an animal model of multiple sclerosis (MS). Congenic rats expressing lower levels of Mcl and Mincle on myeloid cells exhibited a drastic reduction in EAE incidence. In vivo silencing of Mcl and Mincle or blockade of their endogenous ligand SAP130 revealed that these receptors expression in the central nervous system is crucial for T cell recruitment and reactivation into a pathogenic Th17/GM-CSF phenotype. Consistent with this, we uncovered MCL- and MINCLE-expressing cells in brain lesions of MS patients and we further found an upregulation of the MCL/MINCLE signaling pathway and an increased response following MCL/MINCLE stimulation in peripheral blood mononuclear cells from MS ...
How is inbred Dahl salt-sensitive abbreviated? S/JR stands for inbred Dahl salt-sensitive. S/JR is defined as inbred Dahl salt-sensitive rarely.