Clarity or sharpness of OCULAR VISION or the ability of the eye to see fine details. Visual acuity depends on the functions of RETINA, neuronal transmission, and the interpretative ability of the brain. Normal visual acuity is expressed as 20/20 indicating that one can see at 20 feet what should normally be seen at that distance. Visual acuity can also be influenced by brightness, color, and contrast.
A series of tests used to assess various functions of the eyes.
Visual impairments limiting one or more of the basic functions of the eye: visual acuity, dark adaptation, color vision, or peripheral vision. These may result from EYE DISEASES; OPTIC NERVE DISEASES; VISUAL PATHWAY diseases; OCCIPITAL LOBE diseases; OCULAR MOTILITY DISORDERS; and other conditions (From Newell, Ophthalmology: Principles and Concepts, 7th ed, p132).
The electric response evoked in the cerebral cortex by visual stimulation or stimulation of the visual pathways.
The total area or space visible in a person's peripheral vision with the eye looking straightforward.
Area of the OCCIPITAL LOBE concerned with the processing of visual information relayed via VISUAL PATHWAYS.
The selecting and organizing of visual stimuli based on the individual's past experience.
A nonspecific term referring to impaired vision. Major subcategories include stimulus deprivation-induced amblyopia and toxic amblyopia. Stimulus deprivation-induced amblyopia is a developmental disorder of the visual cortex. A discrepancy between visual information received by the visual cortex from each eye results in abnormal cortical development. STRABISMUS and REFRACTIVE ERRORS may cause this condition. Toxic amblyopia is a disorder of the OPTIC NERVE which is associated with ALCOHOLISM, tobacco SMOKING, and other toxins and as an adverse effect of the use of some medications.
Set of cell bodies and nerve fibers conducting impulses from the eyes to the cerebral cortex. It includes the RETINA; OPTIC NERVE; optic tract; and geniculocalcarine tract.
A pair of ophthalmic lenses in a frame or mounting which is supported by the nose and ears. The purpose is to aid or improve vision. It does not include goggles or nonprescription sun glasses for which EYE PROTECTIVE DEVICES is available.
The removal of a cataractous CRYSTALLINE LENS from the eye.
Deviations from the average or standard indices of refraction of the eye through its dioptric or refractive apparatus.
Vision considered to be inferior to normal vision as represented by accepted standards of acuity, field of vision, or motility. Low vision generally refers to visual disorders that are caused by diseases that cannot be corrected by refraction (e.g., MACULAR DEGENERATION; RETINITIS PIGMENTOSA; DIABETIC RETINOPATHY, etc.).
Visualization of a vascular system after intravenous injection of a fluorescein solution. The images may be photographed or televised. It is used especially in studying the retinal and uveal vasculature.
Partial or complete opacity on or in the lens or capsule of one or both eyes, impairing vision or causing blindness. The many kinds of cataract are classified by their morphology (size, shape, location) or etiology (cause and time of occurrence). (Dorland, 27th ed)
Removal of the whole or part of the vitreous body in treating endophthalmitis, diabetic retinopathy, retinal detachment, intraocular foreign bodies, and some types of glaucoma.
Fluid accumulation in the outer layer of the MACULA LUTEA that results from intraocular or systemic insults. It may develop in a diffuse pattern where the macula appears thickened or it may acquire the characteristic petaloid appearance referred to as cystoid macular edema. Although macular edema may be associated with various underlying conditions, it is most commonly seen following intraocular surgery, venous occlusive disease, DIABETIC RETINOPATHY, and posterior segment inflammatory disease. (From Survey of Ophthalmology 2004; 49(5) 470-90)
The ability to detect sharp boundaries (stimuli) and to detect slight changes in luminance at regions without distinct contours. Psychophysical measurements of this visual function are used to evaluate visual acuity and to detect eye disease.
The process in which light signals are transformed by the PHOTORECEPTOR CELLS into electrical signals which can then be transmitted to the brain.
Application of tests and examinations to identify visual defects or vision disorders occurring in specific populations, as in school children, the elderly, etc. It is differentiated from VISION TESTS, which are given to evaluate/measure individual visual performance not related to a specific population.
The inability to see or the loss or absence of perception of visual stimuli. This condition may be the result of EYE DISEASES; OPTIC NERVE DISEASES; OPTIC CHIASM diseases; or BRAIN DISEASES affecting the VISUAL PATHWAYS or OCCIPITAL LOBE.
An imaging method using LASERS that is used for mapping subsurface structure. When a reflective site in the sample is at the same optical path length (coherence) as the reference mirror, the detector observes interference fringes.
Refraction of LIGHT effected by the media of the EYE.
An oval area in the retina, 3 to 5 mm in diameter, usually located temporal to the posterior pole of the eye and slightly below the level of the optic disk. It is characterized by the presence of a yellow pigment diffusely permeating the inner layers, contains the fovea centralis in its center, and provides the best phototropic visual acuity. It is devoid of retinal blood vessels, except in its periphery, and receives nourishment from the choriocapillaris of the choroid. (From Cline et al., Dictionary of Visual Science, 4th ed)
Persons with loss of vision such that there is an impact on activities of daily living.
An area approximately 1.5 millimeters in diameter within the macula lutea where the retina thins out greatly because of the oblique shifting of all layers except the pigment epithelium layer. It includes the sloping walls of the fovea (clivus) and contains a few rods in its periphery. In its center (foveola) are the cones most adapted to yield high visual acuity, each cone being connected to only one ganglion cell. (Cline et al., Dictionary of Visual Science, 4th ed)
Method of measuring and mapping the scope of vision, from central to peripheral of each eye.
The concave interior of the eye, consisting of the retina, the choroid, the sclera, the optic disk, and blood vessels, seen by means of the ophthalmoscope. (Cline et al., Dictionary of Visual Science, 4th ed)
Degenerative changes in the RETINA usually of older adults which results in a loss of vision in the center of the visual field (the MACULA LUTEA) because of damage to the retina. It occurs in dry and wet forms.
Misalignment of the visual axes of the eyes. In comitant strabismus the degree of ocular misalignment does not vary with the direction of gaze. In noncomitant strabismus the degree of misalignment varies depending on direction of gaze or which eye is fixating on the target. (Miller, Walsh & Hoyt's Clinical Neuro-Ophthalmology, 4th ed, p641)
Mental process to visually perceive a critical number of facts (the pattern), such as characters, shapes, displays, or designs.
Unequal curvature of the refractive surfaces of the eye. Thus a point source of light cannot be brought to a point focus on the retina but is spread over a more or less diffuse area. This results from the radius of curvature in one plane being longer or shorter than the radius at right angles to it. (Dorland, 27th ed)
Insertion of an artificial lens to replace the natural CRYSTALLINE LENS after CATARACT EXTRACTION or to supplement the natural lens which is left in place.
Damage or trauma inflicted to the eye by external means. The concept includes both surface injuries and intraocular injuries.
Separation of the inner layers of the retina (neural retina) from the pigment epithelium. Retinal detachment occurs more commonly in men than in women, in eyes with degenerative myopia, in aging and in aphakia. It may occur after an uncomplicated cataract extraction, but it is seen more often if vitreous humor has been lost during surgery. (Dorland, 27th ed; Newell, Ophthalmology: Principles and Concepts, 7th ed, p310-12).
Investigative technique commonly used during ELECTROENCEPHALOGRAPHY in which a series of bright light flashes or visual patterns are used to elicit brain activity.
Diseases affecting the eye.
The administration of substances into the VITREOUS BODY of the eye with a hypodermic syringe.
The use of green light-producing LASERS to stop bleeding. The green light is selectively absorbed by HEMOGLOBIN, thus triggering BLOOD COAGULATION.
The blending of separate images seen by each eye into one composite image.
A condition of an inequality of refractive power of the two eyes.
A refractive error in which rays of light entering the EYE parallel to the optic axis are brought to a focus in front of the RETINA when accommodation (ACCOMMODATION, OCULAR) is relaxed. This results from an overly curved CORNEA or from the eyeball being too long from front to back. It is also called nearsightedness.
A procedure for removal of the crystalline lens in cataract surgery in which an anterior capsulectomy is performed by means of a needle inserted through a small incision at the temporal limbus, allowing the lens contents to fall through the dilated pupil into the anterior chamber where they are broken up by the use of ultrasound and aspirated out of the eye through the incision. (Cline, et al., Dictionary of Visual Science, 4th ed & In Focus 1993;1(1):1)
The absence or restriction of the usual external sensory stimuli to which the individual responds.
Artificial implanted lenses.
The ten-layered nervous tissue membrane of the eye. It is continuous with the OPTIC NERVE and receives images of external objects and transmits visual impulses to the brain. Its outer surface is in contact with the CHOROID and the inner surface with the VITREOUS BODY. The outer-most layer is pigmented, whereas the inner nine layers are transparent.
Disease of the RETINA as a complication of DIABETES MELLITUS. It is characterized by the progressive microvascular complications, such as ANEURYSM, interretinal EDEMA, and intraocular PATHOLOGIC NEOVASCULARIZATION.
Recording of electric potentials in the retina after stimulation by light.
Relatively bright light, or the dazzling sensation of relatively bright light, which produces unpleasantness or discomfort, or which interferes with optimal VISION, OCULAR. (Cline et al., Dictionary of Visual Science, 4th ed)
A noninflammatory, usually bilateral protrusion of the cornea, the apex being displaced downward and nasally. It occurs most commonly in females at about puberty. The cause is unknown but hereditary factors may play a role. The -conus refers to the cone shape of the corneal protrusion. (From Dorland, 27th ed)
A membrane on the vitreal surface of the retina resulting from the proliferation of one or more of three retinal elements: (1) fibrous astrocytes; (2) fibrocytes; and (3) retinal pigment epithelial cells. Localized epiretinal membranes may occur at the posterior pole of the eye without clinical signs or may cause marked loss of vision as a result of covering, distorting, or detaching the fovea centralis. Epiretinal membranes may cause vascular leakage and secondary retinal edema. In younger individuals some membranes appear to be developmental in origin and occur in otherwise normal eyes. The majority occur in association with retinal holes, ocular concussions, retinal inflammation, or after ocular surgery. (Newell, Ophthalmology: Principles and Concepts, 7th ed, p291)
Perforations through the whole thickness of the retina including the macula as the result of inflammation, trauma, degeneration, etc. The concept includes retinal breaks, tears, dialyses, and holes.
Deeply perforating or puncturing type intraocular injuries.
The measurement of curvature and shape of the anterior surface of the cornea using techniques such as keratometry, keratoscopy, photokeratoscopy, profile photography, computer-assisted image processing and videokeratography. This measurement is often applied in the fitting of contact lenses and in diagnosing corneal diseases or corneal changes including keratoconus, which occur after keratotomy and keratoplasty.
The transparent, semigelatinous substance that fills the cavity behind the CRYSTALLINE LENS of the EYE and in front of the RETINA. It is contained in a thin hyaloid membrane and forms about four fifths of the optic globe.
Images seen by one eye.
Blockage of the RETINAL VEIN. Those at high risk for this condition include patients with HYPERTENSION; DIABETES MELLITUS; ATHEROSCLEROSIS; and other CARDIOVASCULAR DISEASES.
The minimum amount of stimulus energy necessary to elicit a sensory response.
A pathological process consisting of the formation of new blood vessels in the CHOROID.
Nystagmus present at birth or caused by lesions sustained in utero or at the time of birth. It is usually pendular, and is associated with ALBINISM and conditions characterized by early loss of central vision. Inheritance patterns may be X-linked, autosomal dominant, or recessive. (Adams et al., Principles of Neurology, 6th ed, p275)
Involuntary movements of the eye that are divided into two types, jerk and pendular. Jerk nystagmus has a slow phase in one direction followed by a corrective fast phase in the opposite direction, and is usually caused by central or peripheral vestibular dysfunction. Pendular nystagmus features oscillations that are of equal velocity in both directions and this condition is often associated with visual loss early in life. (Adams et al., Principles of Neurology, 6th ed, p272)
Diseases of the cornea.
A localized defect in the visual field bordered by an area of normal vision. This occurs with a variety of EYE DISEASES (e.g., RETINAL DISEASES and GLAUCOMA); OPTIC NERVE DISEASES, and other conditions.
An esterified form of TRIAMCINOLONE. It is an anti-inflammatory glucocorticoid used topically in the treatment of various skin disorders. Intralesional, intramuscular, and intra-articular injections are also administered under certain conditions.
Studies in which individuals or populations are followed to assess the outcome of exposures, procedures, or effects of a characteristic, e.g., occurrence of disease.
Evaluation undertaken to assess the results or consequences of management and procedures used in combating disease in order to determine the efficacy, effectiveness, safety, and practicability of these interventions in individual cases or series.
Lenses designed to be worn on the front surface of the eyeball. (UMDNS, 1999)
Defects of color vision are mainly hereditary traits but can be secondary to acquired or developmental abnormalities in the CONES (RETINA). Severity of hereditary defects of color vision depends on the degree of mutation of the ROD OPSINS genes (on X CHROMOSOME and CHROMOSOME 3) that code the photopigments for red, green and blue.
Partial or total replacement of all layers of a central portion of the cornea.
A surgical procedure to correct MYOPIA by CORNEAL STROMA subtraction. It involves the use of a microkeratome to make a lamellar dissection of the CORNEA creating a flap with intact CORNEAL EPITHELIUM. After the flap is lifted, the underlying midstroma is reshaped with an EXCIMER LASER and the flap is returned to its original position.
Hereditary, progressive degeneration of the neuroepithelium of the retina characterized by night blindness and progressive contraction of the visual field.
Surgery performed on the eye or any of its parts.
Studies used to test etiologic hypotheses in which inferences about an exposure to putative causal factors are derived from data relating to characteristics of persons under study or to events or experiences in their past. The essential feature is that some of the persons under study have the disease or outcome of interest and their characteristics are compared with those of unaffected persons.
The aperture in the iris through which light passes.
An ocular disease, occurring in many forms, having as its primary characteristics an unstable or a sustained increase in the intraocular pressure which the eye cannot withstand without damage to its structure or impairment of its function. The consequences of the increased pressure may be manifested in a variety of symptoms, depending upon type and severity, such as excavation of the optic disk, hardness of the eyeball, corneal anesthesia, reduced visual acuity, seeing of colored halos around lights, disturbed dark adaptation, visual field defects, and headaches. (Dictionary of Visual Science, 4th ed)
Agents that dilate the pupil. They may be either sympathomimetics or parasympatholytics.
The positioning and accommodation of eyes that allows the image to be brought into place on the FOVEA CENTRALIS of each eye.
A surgical specialty concerned with the structure and function of the eye and the medical and surgical treatment of its defects and diseases.
Gas lasers with excited dimers (i.e., excimers) as the active medium. The most commonly used are rare gas monohalides (e.g., argon fluoride, xenon chloride). Their principal emission wavelengths are in the ultraviolet range and depend on the monohalide used (e.g., 193 nm for ArF, 308 nm for Xe Cl). These lasers are operated in pulsed and Q-switched modes and used in photoablative decomposition involving actual removal of tissue. (UMDNS, 2005)
Examination of the interior of the eye with an ophthalmoscope.
Hemorrhage into the VITREOUS BODY.
Voluntary or reflex-controlled movements of the eye.
A refractive error in which rays of light entering the eye parallel to the optic axis are brought to a focus behind the retina, as a result of the eyeball being too short from front to back. It is also called farsightedness because the near point is more distant than it is in emmetropia with an equal amplitude of accommodation. (Dorland, 27th ed)
The normal decreasing elasticity of the crystalline lens that leads to loss of accommodation.
The awareness of the spatial properties of objects; includes physical space.
Perception of three-dimensionality.
A type of refractive surgery of the CORNEA to correct MYOPIA and ASTIGMATISM. An EXCIMER LASER is used directly on the surface of the EYE to remove some of the CORNEAL EPITHELIUM thus reshaping the anterior curvature of the cornea.
The science dealing with the correlation of the physical characteristics of a stimulus, e.g., frequency or intensity, with the response to the stimulus, in order to assess the psychologic factors involved in the relationship.
The administration of substances into the eye with a hypodermic syringe.
Observation of a population for a sufficient number of persons over a sufficient number of years to generate incidence or mortality rates subsequent to the selection of the study group.
Normal nystagmus produced by looking at objects moving across the field of vision.
An operation for retinal detachment which reduces the size of the globe by indenting the sclera so that it approximates the retina.
The use of an aberrometer to measure eye tissue imperfections or abnormalities based on the way light passes through the eye which affects the ability of the eye to focus properly.
Disorder occurring in the central or peripheral area of the cornea. The usual degree of transparency becomes relatively opaque.
Suppurative inflammation of the tissues of the internal structures of the eye frequently associated with an infection.
An objective determination of the refractive state of the eye (NEARSIGHTEDNESS; FARSIGHTEDNESS; ASTIGMATISM). By using a RETINOSCOPE, the amount of correction and the power of lens needed can be determined.
Presence of an intraocular lens after cataract extraction.
Atrophy of the optic disk which may be congenital or acquired. This condition indicates a deficiency in the number of nerve fibers which arise in the RETINA and converge to form the OPTIC DISK; OPTIC NERVE; OPTIC CHIASM; and optic tracts. GLAUCOMA; ISCHEMIA; inflammation, a chronic elevation of intracranial pressure, toxins, optic nerve compression, and inherited conditions (see OPTIC ATROPHIES, HEREDITARY) are relatively common causes of this condition.
Bleeding from the vessels of the retina.
The transparent anterior portion of the fibrous coat of the eye consisting of five layers: stratified squamous CORNEAL EPITHELIUM; BOWMAN MEMBRANE; CORNEAL STROMA; DESCEMET MEMBRANE; and mesenchymal CORNEAL ENDOTHELIUM. It serves as the first refracting medium of the eye. It is structurally continuous with the SCLERA, avascular, receiving its nourishment by permeation through spaces between the lamellae, and is innervated by the ophthalmic division of the TRIGEMINAL NERVE via the ciliary nerves and those of the surrounding conjunctiva which together form plexuses. (Cline et al., Dictionary of Visual Science, 4th ed)
Introduction of substances into the body using a needle and syringe.
Asymmetries in the topography and refractive index of the corneal surface that affect visual acuity.
A surgical procedure or KERATOPLASTY involving selective stripping and replacement of diseased host DESCEMET MEMBRANE and CORNEAL ENDOTHELIUM with a suitable and healthy donor posterior lamella. The advantage to this procedure is that the normal corneal surface of the recipient is retained, thereby avoiding corneal surface incisions and sutures.
A form of RETINAL DEGENERATION in which abnormal CHOROIDAL NEOVASCULARIZATION occurs under the RETINA and MACULA LUTEA, causing bleeding and leaking of fluid. This leads to bulging and or lifting of the macula and the distortion or destruction of central vision.
The sensory discrimination of a pattern shape or outline.
The act of knowing or the recognition of a distance by recollective thought, or by means of a sensory process which is under the influence of set and of prior experience.
Inflammation of part or all of the uvea, the middle (vascular) tunic of the eye, and commonly involving the other tunics (sclera and cornea, and the retina). (Dorland, 27th ed)
The real or apparent movement of objects through the visual field.
Disorders of the choroid including hereditary choroidal diseases, neoplasms, and other abnormalities of the vascular layer of the uvea.
The inner portion of a retinal rod or a cone photoreceptor cell, situated between the PHOTORECEPTOR CONNECTING CILIUM and the synapse with the adjacent neurons (RETINAL BIPOLAR CELLS; RETINAL HORIZONTAL CELLS). The inner segment contains the cell body, the nucleus, the mitochondria, and apparatus for protein synthesis.
Inflammation of the optic nerve. Commonly associated conditions include autoimmune disorders such as MULTIPLE SCLEROSIS, infections, and granulomatous diseases. Clinical features include retro-orbital pain that is aggravated by eye movement, loss of color vision, and contrast sensitivity that may progress to severe visual loss, an afferent pupillary defect (Marcus-Gunn pupil), and in some instances optic disc hyperemia and swelling. Inflammation may occur in the portion of the nerve within the globe (neuropapillitis or anterior optic neuritis) or the portion behind the globe (retrobulbar neuritis or posterior optic neuritis).
The pressure of the fluids in the eye.
The dioptric adjustment of the EYE (to attain maximal sharpness of retinal imagery for an object of regard) referring to the ability, to the mechanism, or to the process. Ocular accommodation is the effecting of refractive changes by changes in the shape of the CRYSTALLINE LENS. Loosely, it refers to ocular adjustments for VISION, OCULAR at various distances. (Cline et al., Dictionary of Visual Science, 4th ed)
Sudden ISCHEMIA in the RETINA due to blocked blood flow through the CENTRAL RETINAL ARTERY or its branches leading to sudden complete or partial loss of vision, respectively, in the eye.
The light sensitive outer portion of a retinal rod or a cone photoreceptor cell. The outer segment contains a stack of disk membranes laden with photoreceptive pigments (RETINAL PIGMENTS). The outer segment is connected to the inner segment by a PHOTORECEPTOR CONNECTING CILIUM.
The period following a surgical operation.
The coagulation of tissue by an intense beam of light, including laser (LASER COAGULATION). In the eye it is used in the treatment of retinal detachments, retinal holes, aneurysms, hemorrhages, and malignant and benign neoplasms. (Dictionary of Visual Science, 3d ed)
A visual impairment characterized by the accumulation of fluid under the retina through a defect in the retinal pigment epithelium.
Processes and properties of the EYE as a whole or of any of its parts.
Infection by a variety of fungi, usually through four possible mechanisms: superficial infection producing conjunctivitis, keratitis, or lacrimal obstruction; extension of infection from neighboring structures - skin, paranasal sinuses, nasopharynx; direct introduction during surgery or accidental penetrating trauma; or via the blood or lymphatic routes in patients with underlying mycoses.
Inflammation of the pars plana, ciliary body, and adjacent structures.
The time from the onset of a stimulus until a response is observed.
Agents and endogenous substances that antagonize or inhibit the development of new blood vessels.
Artificial device such as an externally-worn camera attached to a stimulator on the RETINA, OPTIC NERVE, or VISUAL CORTEX, intended to restore or amplify vision.
Focusing on certain aspects of current experience to the exclusion of others. It is the act of heeding or taking notice or concentrating.
The thin, highly vascular membrane covering most of the posterior of the eye between the RETINA and SCLERA.
Conditions which produce injury or dysfunction of the second cranial or optic nerve, which is generally considered a component of the central nervous system. Damage to optic nerve fibers may occur at or near their origin in the retina, at the optic disk, or in the nerve, optic chiasm, optic tract, or lateral geniculate nuclei. Clinical manifestations may include decreased visual acuity and contrast sensitivity, impaired color vision, and an afferent pupillary defect.
The use of photothermal effects of LASERS to coagulate, incise, vaporize, resect, dissect, or resurface tissue.
Inflammation of the choroid as well as the retina and vitreous body. Some form of visual disturbance is usually present. The most important characteristics of posterior uveitis are vitreous opacities, choroiditis, and chorioretinitis.
Type of vision test used to determine COLOR VISION DEFECTS.
Elements of limited time intervals, contributing to particular results or situations.
Loss of epithelial tissue from the surface of the cornea due to progressive erosion and necrosis of the tissue; usually caused by bacterial, fungal, or viral infection.
Methods and procedures for the diagnosis of diseases of the eye or of vision disorders.
Adjustment of the eyes under conditions of low light. The sensitivity of the eye to light is increased during dark adaptation.
Differential response to different stimuli.
Inanimate objects that become enclosed in the eye.
Benign and malignant neoplasms that arise from the optic nerve or its sheath. OPTIC NERVE GLIOMA is the most common histologic type. Optic nerve neoplasms tend to cause unilateral visual loss and an afferent pupillary defect and may spread via neural pathways to the brain.
Partial or total replacement of the CORNEA from one human or animal to another.
Function of the human eye that is used in bright illumination or in daylight (at photopic intensities). Photopic vision is performed by the three types of RETINAL CONE PHOTORECEPTORS with varied peak absorption wavelengths in the color spectrum (from violet to red, 400 - 700 nm).
Diseases, dysfunctions, or disorders of or located in the iris.
Devices that help people with impaired sensory responses.
Incomplete rupture of the zonule with the displaced lens remaining behind the pupil. In dislocation, or complete rupture, the lens is displaced forward into the anterior chamber or backward into the vitreous body. When congenital, this condition is known as ECTOPIA LENTIS.
Photosensitive afferent neurons located primarily within the FOVEA CENTRALIS of the MACULA LUTEA. There are three major types of cone cells (red, blue, and green) whose photopigments have different spectral sensitivity curves. Retinal cone cells operate in daylight vision (at photopic intensities) providing color recognition and central visual acuity.
Swelling of the OPTIC DISK, usually in association with increased intracranial pressure, characterized by hyperemia, blurring of the disk margins, microhemorrhages, blind spot enlargement, and engorgement of retinal veins. Chronic papilledema may cause OPTIC ATROPHY and visual loss. (Miller et al., Clinical Neuro-Ophthalmology, 4th ed, p175)
Bleeding in the anterior chamber of the eye.
Organic siloxanes which are polymerized to the oily stage. The oils have low surface tension and density less than 1. They are used in industrial applications and in the treatment of retinal detachment, complicated by proliferative vitreoretinopathy.
Albinism affecting the eye in which pigment of the hair and skin is normal or only slightly diluted. The classic type is X-linked (Nettleship-Falls), but an autosomal recessive form also exists. Ocular abnormalities may include reduced pigmentation of the iris, nystagmus, photophobia, strabismus, and decreased visual acuity.
The functional superiority and preferential use of one eye over the other. The term is usually applied to superiority in sighting (VISUAL PERCEPTION) or motor task but not difference in VISUAL ACUITY or dysfunction of one of the eyes. Ocular dominance can be modified by visual input and NEUROTROPHIC FACTORS.
A form of ocular misalignment characterized by an excessive convergence of the visual axes, resulting in a "cross-eye" appearance. An example of this condition occurs when paralysis of the lateral rectus muscle causes an abnormal inward deviation of one eye on attempted gaze.
A glucocorticoid given, as the free alcohol or in esterified form, orally, intramuscularly, by local injection, by inhalation, or applied topically in the management of various disorders in which corticosteroids are indicated. (From Martindale, The Extra Pharmacopoeia, 30th ed, p739)
Antibodies from non-human species whose protein sequences have been modified to make them nearly identical with human antibodies. If the constant region and part of the variable region are replaced, they are called humanized. If only the constant region is modified they are called chimeric. INN names for humanized antibodies end in -zumab.
Recording of the average amplitude of the resting potential arising between the cornea and the retina in light and dark adaptation as the eyes turn a standard distance to the right and the left. The increase in potential with light adaptation is used to evaluate the condition of the retinal pigment epithelium.
Infections in the inner or external eye caused by microorganisms belonging to several families of bacteria. Some of the more common genera found are Haemophilus, Neisseria, Staphylococcus, Streptococcus, and Chlamydia.
The organ of sight constituting a pair of globular organs made up of a three-layered roughly spherical structure specialized for receiving and responding to light.
Pathologic processes that affect patients after a surgical procedure. They may or may not be related to the disease for which the surgery was done, and they may or may not be direct results of the surgery.
A glucocorticoid derivative used topically in the treatment of various skin disorders. It is usually employed as a cream, gel, lotion, or ointment. It has also been used topically in the treatment of inflammatory eye, ear, and nose disorders. (From Martindale, The Extra Pharmacopoeia, 30th ed, p732)
The professional practice of primary eye and vision care that includes the measurement of visual refractive power and the correction of visual defects with lenses or glasses.
Function of the human eye that is used in dim illumination (scotopic intensities) or at nighttime. Scotopic vision is performed by RETINAL ROD PHOTORECEPTORS with high sensitivity to light and peak absorption wavelength at 507 nm near the blue end of the spectrum.
The coordination of a sensory or ideational (cognitive) process and a motor activity.
A procedure to surgically correct REFRACTIVE ERRORS by cutting radial slits into the CORNEA to change its refractive properties.
Sterile solutions that are intended for instillation into the eye. It does not include solutions for cleaning eyeglasses or CONTACT LENS SOLUTIONS.
Absence of crystalline lens totally or partially from field of vision, from any cause except after cataract extraction. Aphakia is mainly congenital or as result of LENS DISLOCATION AND SUBLUXATION.
Absence of the crystalline lens resulting from cataract extraction.
Non-invasive method of demonstrating internal anatomy based on the principle that atomic nuclei in a strong magnetic field absorb pulses of radiofrequency energy and emit them as radiowaves which can be reconstructed into computerized images. The concept includes proton spin tomographic techniques.
Partial or complete loss of vision in one half of the visual field(s) of one or both eyes. Subtypes include altitudinal hemianopsia, characterized by a visual defect above or below the horizontal meridian of the visual field. Homonymous hemianopsia refers to a visual defect that affects both eyes equally, and occurs either to the left or right of the midline of the visual field. Binasal hemianopsia consists of loss of vision in the nasal hemifields of both eyes. Bitemporal hemianopsia is the bilateral loss of vision in the temporal fields. Quadrantanopsia refers to loss of vision in one quarter of the visual field in one or both eyes.
Exudates are fluids, CELLS, or other cellular substances that are slowly discharged from BLOOD VESSELS usually from inflamed tissues. Transudates are fluids that pass through a membrane or squeeze through tissue or into the EXTRACELLULAR SPACE of TISSUES. Transudates are thin and watery and contain few cells or PROTEINS.
The thin noncellular outer covering of the CRYSTALLINE LENS composed mainly of COLLAGEN TYPE IV and GLYCOSAMINOGLYCANS. It is secreted by the embryonic anterior and posterior epithelium. The embryonic posterior epithelium later disappears.
A tricarbocyanine dye that is used diagnostically in liver function tests and to determine blood volume and cardiac output.
The illumination of an environment and the arrangement of lights to achieve an effect or optimal visibility. Its application is in domestic or in public settings and in medical and non-medical environments.
Pieces of glass or other transparent materials used for magnification or increased visual acuity.
The surgical removal of the eyeball leaving the eye muscles and remaining orbital contents intact.
The 2nd cranial nerve which conveys visual information from the RETINA to the brain. The nerve carries the axons of the RETINAL GANGLION CELLS which sort at the OPTIC CHIASM and continue via the OPTIC TRACTS to the brain. The largest projection is to the lateral geniculate nuclei; other targets include the SUPERIOR COLLICULI and the SUPRACHIASMATIC NUCLEI. Though known as the second cranial nerve, it is considered part of the CENTRAL NERVOUS SYSTEM.
Inflammation in which both the anterior and posterior segments of the uvea are involved and a specific focus is not apparent. It is often severe and extensive and a serious threat to vision. Causes include systemic diseases such as tuberculosis, sarcoidosis, and syphilis, as well as malignancies. The intermediate segment of the eye is not involved.
The X-shaped structure formed by the meeting of the two optic nerves. At the optic chiasm the fibers from the medial part of each retina cross to project to the other side of the brain while the lateral retinal fibers continue on the same side. As a result each half of the brain receives information about the contralateral visual field from both eyes.
The statistical reproducibility of measurements (often in a clinical context), including the testing of instrumentation or techniques to obtain reproducible results. The concept includes reproducibility of physiological measurements, which may be used to develop rules to assess probability or prognosis, or response to a stimulus; reproducibility of occurrence of a condition; and reproducibility of experimental results.
A group of CORTICOSTEROIDS that affect carbohydrate metabolism (GLUCONEOGENESIS, liver glycogen deposition, elevation of BLOOD SUGAR), inhibit ADRENOCORTICOTROPIC HORMONE secretion, and possess pronounced anti-inflammatory activity. They also play a role in fat and protein metabolism, maintenance of arterial blood pressure, alteration of the connective tissue response to injury, reduction in the number of circulating lymphocytes, and functioning of the central nervous system.
The point or frequency at which all flicker of an intermittent light stimulus disappears.
Imaging techniques used to colocalize sites of brain functions or physiological activity with brain structures.
The adjustment of the eye to variations in the intensity of light. Light adaptation is the adjustment of the eye when the light threshold is increased; DARK ADAPTATION when the light is greatly reduced. (From Cline et al., Dictionary of Visual Science, 4th ed)
Method of making images on a sensitized surface by exposure to light or other radiant energy.
Glial cell derived tumors arising from the optic nerve, usually presenting in childhood.
Ischemic injury to the OPTIC NERVE which usually affects the OPTIC DISK (optic neuropathy, anterior ischemic) and less frequently the retrobulbar portion of the nerve (optic neuropathy, posterior ischemic). The injury results from occlusion of arterial blood supply which may result from TEMPORAL ARTERITIS; ATHEROSCLEROSIS; COLLAGEN DISEASES; EMBOLISM; DIABETES MELLITUS; and other conditions. The disease primarily occurs in the sixth decade or later and presents with the sudden onset of painless and usually severe monocular visual loss. Anterior ischemic optic neuropathy also features optic disk edema with microhemorrhages. The optic disk appears normal in posterior ischemic optic neuropathy. (Glaser, Neuro-Ophthalmology, 2nd ed, p135)
A scientific tool based on ULTRASONOGRAPHY and used not only for the observation of microstructure in metalwork but also in living tissue. In biomedical application, the acoustic propagation speed in normal and abnormal tissues can be quantified to distinguish their tissue elasticity and other properties.
Tumors of the choroid; most common intraocular tumors are malignant melanomas of the choroid. These usually occur after puberty and increase in incidence with advancing age. Most malignant melanomas of the uveal tract develop from benign melanomas (nevi).
Abnormal sensitivity to light. This may occur as a manifestation of EYE DISEASES; MIGRAINE; SUBARACHNOID HEMORRHAGE; MENINGITIS; and other disorders. Photophobia may also occur in association with DEPRESSION and other MENTAL DISORDERS.
Measurement of distances or movements by means of the phenomena caused by the interference of two rays of light (optical interferometry) or of sound (acoustic interferometry).
Photosensitive protein complexes of varied light absorption properties which are expressed in the PHOTORECEPTOR CELLS. They are OPSINS conjugated with VITAMIN A-based chromophores. Chromophores capture photons of light, leading to the activation of opsins and a biochemical cascade that ultimately excites the photoreceptor cells.
An exudate between the RETINA and CHOROID from various sources including the vitreous cavity, SUBARACHNOID SPACE, or abnormal vessels.
An excessive amount of fluid in the cornea due to damage of the epithelium or endothelium causing decreased visual acuity.
That portion of the electromagnetic spectrum in the visible, ultraviolet, and infrared range.
Transmission of gene defects or chromosomal aberrations/abnormalities which are expressed in extreme variation in the structure or function of the eye. These may be evident at birth, but may be manifested later with progression of the disorder.
Glaucoma in which the angle of the anterior chamber is open and the trabecular meshwork does not encroach on the base of the iris.
An abrupt voluntary shift in ocular fixation from one point to another, as occurs in reading.
Conditions which affect the structure or function of the pupil of the eye, including disorders of innervation to the pupillary constrictor or dilator muscles, and disorders of pupillary reflexes.
Signals for an action; that specific portion of a perceptual field or pattern of stimuli to which a subject has learned to respond.
Neurons of the innermost layer of the retina, the internal plexiform layer. They are of variable sizes and shapes, and their axons project via the OPTIC NERVE to the brain. A small subset of these cells act as photoreceptors with projections to the SUPRACHIASMATIC NUCLEUS, the center for regulating CIRCADIAN RHYTHM.
Inflammation of the cornea.
Therapy using oral or topical photosensitizing agents with subsequent exposure to light.
Cognitive disorders characterized by an impaired ability to perceive the nature of objects or concepts through use of the sense organs. These include spatial neglect syndromes, where an individual does not attend to visual, auditory, or sensory stimuli presented from one side of the body.
Failure or imperfection of vision at night or in dim light, with good vision only on bright days. (Dorland, 27th ed)

Zernike representation of corneal topography height data after nonmechanical penetrating keratoplasty. (1/5631)

PURPOSE: To demonstrate a mathematical method for decomposition of discrete corneal topography height data into a set of Zernike polynomials and to demonstrate the clinical applicability of these computations in the postkeratoplasty cornea. METHODS: Fifty consecutive patients with either Fuchs' dystrophy (n = 20) or keratoconus (n = 30) were seen at 3 months, 6 months, and 1 year (before suture removal) and again after suture removal following nonmechanical trephination with the excimer laser. Patients were assessed using regular keratometry, corneal topography (TMS-1, simulated keratometry [SimK]), subjective refraction, and best-corrected visual acuity (VA) at each interval. A set of Zernike coefficients with radial degree 8 was calculated to fit two model surfaces: a complete representation (TOTAL) and a representation with parabolic terms only to define an approximate spherocylindrical surface (PARABOLIC). The root mean square error (RMS) was calculated comparing the corneal raw height data with TOTAL (TOTALRMS) and PARABOLIC (PARABOLICRMS). The cylinder of subjective refraction was correlated with the keratometric readings, the SimK, and the respective Zernike parameter. Visual acuity was correlated with the tilt components of the Zernike expansion. RESULTS: The measured corneal surface could be approximated by the composed surface 1 with TOTALRMS < or = 1.93 microm and by surface 2 with PARABOLICRMS < or = 3.66 microm. Mean keratometric reading after suture removal was 2.8+/-0.6 D. At all follow-up examinations, the SimK yielded higher values, whereas the keratometric reading and the refractive cylinder yielded lower values than the respective Zernike parameter. The correlation of the Zernike representation and the refractive cylinder (P = 0.02 at 3 months, P = 0.05 at 6 months and at 1 year, and P = 0.01 after suture removal) was much better than the correlation of the SimK and refractive cylinder (P = 0.3 at 3 months, P = 0.4 at 6 months, P = 0.2 at 1 year, and P = 0.1 after suture removal). Visual acuity increased from 0.23+/-0.10 at the 3-month evaluation to 0.54+/-0.19 after suture removal. After suture removal, there was a statistically significant inverse correlation between VA and tilt (P = 0.02 in patients with keratoconus and P = 0.05 in those with Fuchs' dystrophy). CONCLUSIONS: Zernike representation of corneal topography height data renders a reconstruction of clinically relevant corneal topography parameters with a marked reduction of redundance and a small error. Correlation of amount/axis of refractive cylinder with respective Zernike parameters is more accurate than with keratometry or respective SimK values of corneal topography analysis.  (+info)

Management of phacolytic glaucoma: experience of 135 cases. (2/5631)

We retrospectively analyzed 135 eyes with phacolytic glaucoma. A trabeculectomy was added to standard cataract surgery if symptoms endured for more than seven days, or if preoperative control of intraocular pressure (IOP) with maximal medical treatment was inadequate. In the early postoperative period, IOP was significantly lower in the combined surgery group (89 eyes) compared to the cataract surgery group (46 eyes) (p < 0.001). At 6 months there was no difference in IOP or visual acuity between the two groups. There were no serious complications related to trabeculectomy. It is reasonable to conclude that in eyes with a long duration of phacolytic glaucoma, addition of a trabeculectomy to cataract surgery is safe, prevents postoperative rise in intraocular pressure and decreases the need for systemic hypotensive medications. A randomized trial is on to further address this question.  (+info)

Effect of pilocarpine on visual acuity and on the dimensions of the cornea and anterior chamber. (3/5631)

The effect of pilocarpine on visual acuity and on the dimensions of the cornea, anterior chamber, and lens were studied in two groups of subjects. Significant changes in ocular tension, corneal curvature, anterior chamber depth, and lens anterior radius were found in a group of 55 glaucomatous eyes as a result of pilocarpine treatment, but there was no change in corneal thickness. Out of 102 glaucomatous eyes 78 became relatively myopic, and this appears to be due to changes in the dimensions of the lens of the eye similar to those occurring in accommodation, as a result of the effect of the drug on the ciliary muscle. The effect of pilocarpine on anterior chamber depth, area, and volume was studied in 125 eyes using a photographic method, and a significant reduction in the dimensions of the anterior chamber was observed as a result of the administration of pilocarpine. A significant correlation between depth and volume was also noted and the implications of this are discussed.  (+info)

De novo lesions in presumed ocular histoplasmosis-like syndrome. (4/5631)

Two patients with multifocal choroiditis similar or identical to POHS are presented. Colour photographs and fluorescein angiography document the occurrence of de novo lesions in the originally involved eye. The cases also demonstrate the development of new choroidal lesions within the originally involved eye, the early evolution of the "basic choroidal lesion", and the need for fluorescein angiography for visualizing the underlying choroidal lesion.  (+info)

Prognosis of perforating eye injury. (5/5631)

The assessment of visual function in a series of 130 consecutive patients of perforating eye injuries, revealed that visual acuity of 6/12 or better was regained in 63 per cent, between 6/60 and 6/18 in 9-2 per cent, less than 6/60 in 15-3 per cent, and enucleation was necessary in 9-2 per cent. In 3 per cent, the eyes were retained as blind, symptomfree, and cosmetically satisfactory organs. Two eyes were found to develop complete traumatic aniridia. None in the series was found to have sympathetic ophthalmitis.  (+info)

Vitrectomy in 125 eyes with diabetic vitreous haemorrhage. (6/5631)

A total of 125 consecutive eyes, all registered blind with diabetic vitreous haemorrhage, underwent pars plana vitrectomy with the vitrophage. Sixty-six per cent experienced some improvement in their visual acuity; 24 per cent were unchanged and 10 per cent were worse postoperatively. The major surgical complication was controllable haemorrhage (23 per cent). No retinal dialysis occurred. Significant postoperative complications were transient (71 per cent) and persistent (11 per cent) corneal oedema, early (8 per cent) and late (13 per cent) vitreous haemorrhage, transient (30 per cent) and persistent (6 per cent) rise in intraocular pressure, and rubeosis iridis (5 per cent).  (+info)

A prospective study of xenon arc photocoagulation for central retinal vein occlusion. (7/5631)

Twenty patients with central retinal vein occlusion were randomly divided into two groups in a prospective study to evaluate the effects of xenon are photocoagulation in central retinal vein occlusion. The patients in one group were treated with 360 degrees scatter xenon photocoagulation and the others received no treatment. The average follow-up was 18 months. There were no cases of rubeosis or neovascular glaucoma in the treated group. Two patients in the untreated group developed rubeosis with subsequent neovascular glaucoma. There was no significant difference in the visual prognosis or in fundus neovascularization between the groups.  (+info)

Characteristics of discrepancies between self-reported visual function and measured reading speed. Salisbury Eye Evaluation Project Team. (8/5631)

PURPOSE: Visual impairment is a risk factor for morbidity in the elderly and is often screened for by self-report. This study evaluates whether there are subsets for whom there is a discrepancy between self-reported and measured function. METHODS: The prevalence of a discrepancy between self-reported difficulty reading a newspaper and measured reading speed was determined in 2520 community-based men and women, aged 65 to 84 years, and the discrepant group characterized by polychotomous regression. RESULTS: Of subjects who reported minimal difficulty reading a newspaper, 10.8% (227/2107) read newsprint-sized text (0.21 degrees) more slowly than 80 words/min, a level previously shown to be necessary for sustained reading. Poor visual acuity, presence of psychiatric symptoms, and less satisfaction with vision were associated with being in the group that read slowly and reported difficulty with reading. Better cognition, better visual acuity, more years of education, white race, and fewer psychiatric symptoms were associated with being in the group that read more quickly and reported minimal difficulty. When reading the text size at which subjects read their fastest, only 2.6% of those with minimal difficulty remained discrepant. These individuals were more likely to have less education, be male, be African American, and have poorer cognitive status than those who did not remain discrepant. CONCLUSIONS: A subset of the elderly population have a substantial discrepancy between self-reported reading difficulty and measured reading speed. In some, this discrepancy may be based on underlying expectations and experiences, and in others it may represent a transition from no visual impairment to visual impairment.  (+info)

Some common types of vision disorders include:

1. Myopia (nearsightedness): A condition where close objects are seen clearly, but distant objects appear blurry.
2. Hyperopia (farsightedness): A condition where distant objects are seen clearly, but close objects appear blurry.
3. Astigmatism: A condition where the cornea or lens of the eye is irregularly shaped, causing blurred vision at all distances.
4. Presbyopia: A condition that occurs as people age, where the lens of the eye loses flexibility and makes it difficult to focus on close objects.
5. Amblyopia (lazy eye): A condition where one eye has reduced vision due to abnormal development or injury.
6. Strabismus (crossed eyes): A condition where the eyes are misaligned and point in different directions.
7. Color blindness: A condition where people have difficulty perceiving certain colors, usually red and green.
8. Retinal disorders: Conditions that affect the retina, such as age-related macular degeneration, diabetic retinopathy, or retinal detachment.
9. Glaucoma: A group of conditions that damage the optic nerve, often due to increased pressure in the eye.
10. Cataracts: A clouding of the lens in the eye that can cause blurred vision and sensitivity to light.

Vision disorders can be diagnosed through a comprehensive eye exam, which includes a visual acuity test, refraction test, and dilated eye exam. Treatment options for vision disorders depend on the specific condition and may include glasses or contact lenses, medication, surgery, or a combination of these.

The amblyopic eye may have reduced visual sharpness and/or abnormal ocular alignment (strabismus). The other eye is generally normal or has better vision. Amblyopia is often present at birth but may not be noticed until the child is a few years old. It can also result from various conditions, such as strabismus, cataracts, or differences in the refractive error of the two eyes (anisometropic amblyopia).

The most common form of amblyopia is anisometropic amblyopia, which occurs when there is a significant difference in the refractive power between the two eyes. This can cause the brain to favor one eye over the other, leading to reduced vision in the amblyopic eye. Amblyopia can be treated with glasses or contact lenses, patching the better eye to force the weaker eye to work harder, or surgery to correct strabismus or anisometropia.

Early detection and treatment are important to prevent long-term visual impairment. However, amblyopia can sometimes persist even after treatment, and it is a leading cause of monocular vision in adults.

Myopia occurs when the eyeball is too long or the cornea is too steep, causing light to focus in front of the retina instead of directly on it. Hyperopia is the opposite, where the eyeball is too short or the cornea is too flat, causing light to focus behind the retina. Astigmatism is caused by an irregularly shaped cornea, which causes light to focus at multiple points instead of one. Presbyopia is a loss of near vision that occurs as people age, making it harder to see close objects clearly.

In addition to these common refractive errors, there are other, less common conditions that can affect the eyes and cause blurred vision, such as amblyopia (lazy eye), strabismus (crossed eyes), and retinal detachment. These conditions can be caused by a variety of factors, including genetics, injury, or disease.

Refractive errors can have a significant impact on daily life, affecting everything from work and school performance to social interactions and overall quality of life. Fortunately, with the help of corrective lenses or surgery, many people are able to achieve clear vision and lead fulfilling lives.

Low vision is not the same as blindness, but it does affect an individual's ability to perform daily activities such as reading, driving, and recognizing faces. The condition can be treated with low vision aids such as specialized glasses, telescopes, and video magnifiers that enhance visual acuity and improve the ability to see objects and details more clearly.

In the medical field, Low Vision is often used interchangeably with the term "visual impairment" which refers to any degree of vision loss that cannot be corrected by regular glasses or contact lenses. Visual impairment can range from mild to severe and can have a significant impact on an individual's quality of life.

Low Vision is a common condition among older adults, with approximately 20% of people over the age of 65 experiencing some degree of visual impairment. However, Low Vision can also affect younger individuals, particularly those with certain eye conditions such as retinitis pigmentosa or other inherited eye disorders.

Overall, Low Vision is a condition that affects an individual's ability to see clearly and perform daily activities, and it is important for individuals experiencing vision loss to seek medical attention to determine the cause of their symptoms and explore available treatment options.

There are different types of cataracts, including:

1. Nuclear cataract: This is the most common type of cataract and affects the center of the lens.
2. Cortical cataract: This type of cataract affects the outer layer of the lens and can cause a "halo" effect around lights.
3. Posterior subcapsular cataract: This type of cataract affects the back of the lens and is more common in younger people and those with diabetes.
4. Congenital cataract: This type of cataract is present at birth and can be caused by genetic factors or other conditions.

Symptoms of cataracts can include:

* Blurred vision
* Double vision
* Sensitivity to light
* Glare
* Difficulty seeing at night
* Fading or yellowing of colors

Cataracts can be diagnosed with a comprehensive eye exam, which includes a visual acuity test, dilated eye exam, and imaging tests such as ultrasound or optical coherence tomography (OCT).

Treatment for cataracts typically involves surgery to remove the clouded lens and replace it with an artificial one called an intraocular lens (IOL). The type of IOL used will depend on the patient's age, visual needs, and other factors. In some cases, cataracts may be removed using a laser-assisted procedure.

In addition to surgery, there are also non-surgical treatments for cataracts, such as glasses or contact lenses, which can help improve vision. However, these treatments do not cure the underlying condition and are only temporary solutions.

It's important to note that cataracts are a common age-related condition and can affect anyone over the age of 40. Therefore, it's important to have regular eye exams to monitor for any changes in vision and to detect cataracts early on.

In summary, cataracts are a clouding of the lens in the eye that can cause blurred vision, double vision, sensitivity to light, and other symptoms. Treatment typically involves surgery to remove the clouded lens and replace it with an artificial one, but non-surgical treatments such as glasses or contact lenses may also be used. Regular eye exams are important for detecting cataracts early on and monitoring vision health.

Symptoms of macular edema may include blurred vision, distorted vision, blind spots, and sensitivity to light. Diagnosis is typically made through a comprehensive eye exam, including a visual acuity test and imaging tests such as optical coherence tomography (OCT).

Treatment for macular edema depends on the underlying cause of the condition. In some cases, medications such as anti-vascular endothelial growth factor (VEGF) injections or corticosteroids may be prescribed to reduce fluid buildup and swelling in the retina. In more severe cases, surgical intervention may be necessary, such as a vitrectomy to remove the vitreous gel and relieve pressure on the retina.

Prevention of macular edema includes managing underlying conditions such as diabetes and age-related macular degeneration, as well as maintaining regular eye exams to detect and treat any changes in the retina early on. Early detection and treatment can help prevent vision loss from macular edema.

There are different types of blindness, including:

1. Congenital blindness: Blindness that is present at birth, often due to genetic mutations or abnormalities in the development of the eye and brain.
2. Acquired blindness: Blindness that develops later in life due to injury, disease, or other factors.
3. Amblyopia: A condition where one eye has reduced vision due to misalignment or other causes.
4. Glaucoma: A group of eye conditions that can damage the optic nerve and lead to blindness if left untreated.
5. Retinitis pigmentosa: A degenerative disease that affects the retina and can cause blindness.
6. Cataracts: A clouding of the lens in the eye that can impair vision and eventually cause blindness if left untreated.
7. Macular degeneration: A condition where the macula, a part of the retina responsible for central vision, deteriorates and causes blindness.

There are various treatments and therapies for blindness, depending on the underlying cause. These may include medications, surgery, low vision aids, and assistive technology such as braille and audio books, screen readers, and voice-controlled software. Rehabilitation programs can also help individuals adapt to blindness and lead fulfilling lives.

There are two main types of MD:

1. Dry Macular Degeneration (DMD): This is the most common form of MD, accounting for about 90% of cases. It is caused by the gradual accumulation of waste material in the macula, which can lead to cell death and vision loss over time.
2. Wet Macular Degeneration (WMD): This type of MD is less common but more aggressive, accounting for about 10% of cases. It occurs when new blood vessels grow underneath the retina, leaking fluid and causing damage to the macula. This can lead to rapid vision loss if left untreated.

The symptoms of MD can vary depending on the severity and type of the condition. Common symptoms include:

* Blurred vision
* Distorted vision (e.g., straight lines appearing wavy)
* Difficulty reading or recognizing faces
* Difficulty adjusting to bright light
* Blind spots in central vision

MD can have a significant impact on daily life, making it difficult to perform everyday tasks such as driving, reading, and recognizing faces.

There is currently no cure for MD, but there are several treatment options available to slow down the progression of the disease and manage its symptoms. These include:

* Anti-vascular endothelial growth factor (VEGF) injections: These medications can help prevent the growth of new blood vessels and reduce inflammation in the macula.
* Photodynamic therapy: This involves the use of a light-sensitive drug and low-intensity laser to damage and shrink the abnormal blood vessels in the macula.
* Vitamin supplements: Certain vitamins, such as vitamin C, E, and beta-carotene, have been shown to slow down the progression of MD.
* Laser surgery: This can be used to reduce the number of abnormal blood vessels in the macula and improve vision.

It is important for individuals with MD to receive regular monitoring and treatment from an eye care professional to manage their condition and prevent complications.

There are several types of strabismus, including:

* Esotropia: where one eye turns inward toward the nose
* Exotropia: where one eye turns outward away from the face
* Hypertropia: where one eye turns upward
* Hypotropia: where one eye turns downward
* Duane's syndrome: a rare type of strabismus that affects only one eye and is caused by nerve damage.

Strabismus can have both visual and social consequences, including:

* Difficulty with depth perception and binocular vision
* Blurred or double vision
* Difficulty with eye teaming and tracking
* Poor eye-hand coordination
* Social and emotional effects such as low self-esteem, anxiety, and depression.

Treatment options for strabismus include:

* Glasses or contact lenses to correct refractive errors
* Prism lenses to align the eyes
* Eye exercises to strengthen the muscles and improve eye teaming
* Surgery to adjust the position of the muscles that control eye movement.

It is important for individuals with strabismus to receive timely and appropriate treatment to address the underlying cause of the condition and prevent long-term vision loss and social difficulties.

Astigmatism can occur in people of all ages and is usually present at birth, but it may not become noticeable until later in life. It may also develop as a result of an injury or surgery. Astigmatism can be corrected with glasses, contact lenses, or refractive surgery, such as LASIK.

There are different types of astigmatism, including:

1. Corneal astigmatism: This is the most common type of astigmatism and occurs when the cornea is irregularly shaped.
2. Lens astigmatism: This type of astigmatism occurs when the lens inside the eye is irregularly shaped.
3. Mixed astigmatism: This type of astigmatism occurs when both the cornea and lens are irregularly shaped.

Astigmatism can cause a range of symptoms, including:

* Blurred vision at all distances
* Distorted vision (such as seeing objects as being stretched out or blurry)
* Eye strain or fatigue
* Headaches or eye discomfort
* Squinting or tilting the head to see clearly

If you suspect you have astigmatism, it's important to see an eye doctor for a comprehensive eye exam. Astigmatism can be diagnosed with a visual acuity test and a retinoscopy, which measures the way the light enters the eye.

Astigmatism is a common vision condition that can be easily corrected with glasses, contact lenses, or refractive surgery. If you have astigmatism, it's important to seek professional treatment to improve your vision and reduce any discomfort or strain on the eyes.

Types of Eye Injuries:

1. Corneal abrasion: A scratch on the cornea, the clear outer layer of the eye.
2. Conjunctival bleeding: Bleeding in the conjunctiva, the thin membrane that covers the white part of the eye.
3. Hyphema: Blood in the space between the iris and the cornea.
4. Hemorrhage: Bleeding in the eyelid or under the retina.
5. Retinal detachment: Separation of the retina from the underlying tissue, which can cause vision loss if not treated promptly.
6. Optic nerve damage: Damage to the nerve that carries visual information from the eye to the brain, which can cause vision loss or blindness.
7. Orbital injury: Injury to the bones and tissues surrounding the eye, which can cause double vision, swelling, or vision loss.

Symptoms of Eye Injuries:

1. Pain in the eye or around the eye
2. Redness and swelling of the eye or eyelid
3. Difficulty seeing or blurred vision
4. Sensitivity to light
5. Double vision or loss of vision
6. Discharge or crusting around the eye
7. Swelling of the eyelids or face

Treatment of Eye Injuries:

1. Depending on the severity and nature of the injury, treatment may include antibiotics, pain relief medication, or surgery.
2. In some cases, a tube may be inserted into the eye to help drain fluid or prevent pressure from building up.
3. In severe cases, vision may not return completely, but there are many options for corrective glasses and contact lenses to improve remaining vision.
4. It is essential to seek medical attention immediately if there is a foreign object in the eye, as this can cause further damage if left untreated.
5. In cases of penetrating trauma, such as a blow to the eye, it is important to seek medical attention right away, even if there are no immediate signs of injury.
6. Follow-up appointments with an ophthalmologist are essential to monitor healing and address any complications that may arise.

The retina is a layer of cells that lines the inside of the eye and senses light to send visual signals to the brain. When the retina becomes detached, it can no longer function properly, leading to vision loss or distortion.

Retinal detachment can be caused by a variety of factors, including:

1. Age-related changes: As we age, the vitreous gel that fills the eye can become more liquid and pull away from the retina, causing a retinal detachment.
2. Injury or trauma: A blow to the head or a penetrating injury can cause a retinal detachment.
3. Medical conditions: Certain conditions, such as diabetes, high blood pressure, and sickle cell disease, can increase the risk of developing a retinal detachment.
4. Genetic factors: Some people may be more prone to developing a retinal detachment due to inherited genetic factors.

Symptoms of retinal detachment may include:

1. Flashes of light: People may see flashes of light in the peripheral vision.
2. Floaters: Specks or cobwebs may appear in the vision, particularly in the periphery.
3. Blurred vision: Blurred vision or distorted vision may occur as the retina becomes detached.
4. Loss of vision: In severe cases, a retinal detachment can cause a complete loss of vision in one eye.

If you experience any of these symptoms, it is important to seek medical attention immediately. A comprehensive eye exam can diagnose a retinal detachment and determine the appropriate treatment.

Treatment for retinal detachment typically involves surgery to reattach the retina to the underlying tissue. In some cases, laser surgery may be used to seal off any tears or holes in the retina that caused the detachment. In more severe cases, a scleral buckle or other device may be implanted to support the retina and prevent further detachment.

In addition to surgical treatment, there are some lifestyle changes you can make to help reduce your risk of developing a retinal detachment:

1. Quit smoking: Smoking has been linked to an increased risk of retinal detachment. Quitting smoking can help reduce this risk.
2. Maintain a healthy blood pressure: High blood pressure can increase the risk of retinal detachment. Monitoring and controlling your blood pressure can help reduce this risk.
3. Wear protective eyewear: If you participate in activities that could potentially cause eye injury, such as sports or working with hazardous materials, wearing protective eyewear can help reduce the risk of retinal detachment.
4. Get regular eye exams: Regular comprehensive eye exams can help detect any potential issues with the retina before they become serious problems.

Overall, a retinal detachment is a serious condition that requires prompt medical attention to prevent long-term vision loss. By understanding the causes and symptoms of retinal detachment, as well as making lifestyle changes to reduce your risk, you can help protect your vision and maintain good eye health.

There are many different types of eye diseases, including:

1. Cataracts: A clouding of the lens in the eye that can cause blurry vision and blindness.
2. Glaucoma: A group of diseases that damage the optic nerve and can lead to vision loss and blindness.
3. Age-related macular degeneration (AMD): A condition that causes vision loss in older adults due to damage to the macula, the part of the retina responsible for central vision.
4. Diabetic retinopathy: A complication of diabetes that can cause damage to the blood vessels in the retina and lead to vision loss.
5. Detached retina: A condition where the retina becomes separated from the underlying tissue, leading to vision loss.
6. Macular hole: A small hole in the macula that can cause vision loss.
7. Amblyopia (lazy eye): A condition where one eye is weaker than the other and has reduced vision.
8. Strabismus (crossed eyes): A condition where the eyes are not aligned properly and point in different directions.
9. Conjunctivitis: An inflammation of the conjunctiva, the thin membrane that covers the white part of the eye and the inside of the eyelids.
10. Dry eye syndrome: A condition where the eyes do not produce enough tears, leading to dryness, itchiness, and irritation.

Eye diseases can be caused by a variety of factors, including genetics, age, environmental factors, and certain medical conditions. Some eye diseases are inherited, while others are acquired through lifestyle choices or medical conditions.

Symptoms of eye diseases can include blurry vision, double vision, eye pain, sensitivity to light, and redness or inflammation in the eye. Treatment options for eye diseases depend on the specific condition and can range from medication, surgery, or lifestyle changes.

Regular eye exams are important for detecting and managing eye diseases, as many conditions can be treated more effectively if caught early. If you experience any symptoms of eye disease or have concerns about your vision, it is important to see an eye doctor as soon as possible.

Anisometropia is typically diagnosed with a comprehensive eye exam, which includes visual acuity testing, refraction, and retinoscopy. Treatment options for anisometropia depend on the underlying cause and severity of the condition, and may include glasses or contact lenses, prism lenses, or surgery. In some cases, anisometropia can be treated with orthokeratology (OK) or corneal reshaping, which involves wearing a specialized contact lens at night to reshape the cornea and improve vision during the day.

Anisometropia is relatively rare, but it can have a significant impact on quality of life, particularly in children and young adults. If you suspect that you or your child may have anisometropia, it's important to schedule an eye exam as soon as possible to determine the underlying cause and develop an appropriate treatment plan.

Myopia can be caused by a variety of factors, including:

1. Genetics: Myopia can run in families, and people with a family history of myopia are more likely to develop the condition.
2. Near work: Spending too much time doing close-up activities such as reading or using digital devices can increase the risk of developing myopia.
3. Poor posture: Slouching or leaning forward can cause the eye to focus incorrectly, leading to myopia.
4. Nutritional deficiencies: A diet lacking in essential nutrients such as vitamin D and omega-3 fatty acids may contribute to the development of myopia.
5. Eye stress: Prolonged eye strain due to excessive near work or other activities can lead to myopia.

Symptoms of myopia include:

1. Difficulty seeing distant objects clearly
2. Headaches or eye strain from trying to focus on distant objects
3. Squinting or rubbing the eyes to try to see distant objects more clearly
4. Difficulty seeing in low light conditions
5. Blurry vision at a distance, with close objects appearing clear.

Myopia can be diagnosed with a comprehensive eye exam, which includes a visual acuity test, refraction test, and retinoscopy. Treatment options for myopia include:

1. Glasses or contact lenses: These corrective lenses refract light properly onto the retina, allowing clear vision of both close and distant objects.
2. Laser eye surgery: Procedures such as LASIK can reshape the cornea to improve its curvature and reduce myopia.
3. Orthokeratology (ORTHO-K): A non-surgical procedure that uses a specialized contact lens to reshape the cornea while you sleep.
4. Myopia control: This involves using certain treatments or techniques to slow down the progression of myopia in children and young adults.
5. Multifocal lenses: These lenses have multiple focal points, allowing for clear vision of both near and distant objects without the need for glasses or contact lenses.

In conclusion, myopia is a common vision condition that can be caused by a variety of factors and symptoms can include difficulty seeing distant objects clearly, headaches, and eye strain. Treatment options include glasses or contact lenses, laser eye surgery, ORTHO-K, myopia control, and multifocal lenses. It is important to consult an eye doctor for a comprehensive evaluation and to determine the best course of treatment for your specific case of myopia.

There are two main types of DR:

1. Non-proliferative diabetic retinopathy (NPDR): This is the early stage of DR, where the blood vessels in the retina become damaged and start to leak fluid or bleed. The symptoms can be mild or severe and may include blurred vision, floaters, and flashes of light.
2. Proliferative diabetic retinopathy (PDR): This is the advanced stage of DR, where new blood vessels start to grow in the retina. These vessels are weak and can cause severe bleeding, leading to vision loss.

DR is a common complication of diabetes, and it is estimated that up to 80% of people with diabetes will develop some form of DR over their lifetime. The risk of developing DR increases with the duration of diabetes and the level of blood sugar control.

Early detection and treatment of DR can help to prevent vision loss, so it is important for people with diabetes to have regular eye exams to monitor their retinal health. Treatment options for DR include laser surgery, injections of anti-vascular endothelial growth factor (VEGF) medications, and vitrectomy, a surgical procedure to remove the vitreous gel and blood from the eye.

Preventing Diabetic Retinopathy

While there is no surefire way to prevent diabetic retinopathy (DR), there are several steps that people with diabetes can take to reduce their risk of developing this complication:

1. Control blood sugar levels: Keeping blood sugar levels within a healthy range can help to slow the progression of DR. This can be achieved through a combination of diet, exercise, and medication.
2. Monitor blood pressure: High blood pressure can damage the blood vessels in the retina, so it is important to monitor and control blood pressure to reduce the risk of DR.
3. Maintain healthy blood lipids: Elevated levels of low-density lipoprotein (LDL) cholesterol and lower levels of high-density lipoprotein (HDL) cholesterol can increase the risk of DR.
4. Quit smoking: Smoking can damage the blood vessels in the retina and increase the risk of DR.
5. Maintain a healthy weight: Obesity is a risk factor for DR, so maintaining a healthy weight can help to reduce the risk of this complication.
6. Get regular eye exams: Regular eye exams can help to detect DR in its early stages, when it is easier to treat and prevent vision loss.

Preventing Diabetic Retinopathy

While there is no cure for diabetic retinopathy (DR), there are several treatment options available to help manage the condition and prevent vision loss. These include:

1. Laser surgery: This is a common treatment for early-stage DR, where a laser is used to shrink abnormal blood vessels in the retina and reduce the risk of further damage.
2. Injection therapy: Medications such as anti-vascular endothelial growth factor (VEGF) injections can be used to shrink abnormal blood vessels and reduce swelling in the retina.
3. Vitrectomy: In severe cases of DR, a vitrectomy may be performed to remove scar tissue and blood from the center of the eye.
4. Blood pressure control: Maintaining healthy blood pressure can help to slow the progression of DR.
5. Blood glucose control: Keeping blood sugar levels under control can also slow the progression of DR.
6. Follow-up care: Regular follow-up appointments with an eye doctor are important to monitor the progress of DR and adjust treatment as needed.

Early detection and treatment of diabetic retinopathy can help to prevent vision loss and improve outcomes for individuals with this complication of diabetes. By managing blood sugar levels, blood pressure, and cholesterol, and by getting regular eye exams, individuals with diabetes can reduce their risk of developing DR and other diabetic complications.

Examples of retinal diseases include:

1. Age-related macular degeneration (AMD): a leading cause of vision loss in people over the age of 50, AMD affects the macula, the part of the retina responsible for central vision.
2. Diabetic retinopathy (DR): a complication of diabetes that damages blood vessels in the retina and can cause blindness.
3. Retinal detachment: a condition where the retina becomes separated from the underlying tissue, causing vision loss.
4. Macular edema: swelling of the macula that can cause vision loss.
5. Retinal vein occlusion (RVO): a blockage of the small veins in the retina that can cause vision loss.
6. Retinitis pigmentosa (RP): a group of inherited disorders that affect the retina and can cause progressive vision loss.
7. Leber congenital amaurosis (LCA): an inherited disorder that causes blindness or severe visual impairment at birth or in early childhood.
8. Stargardt disease: a rare inherited disorder that affects the retina and can cause progressive vision loss, usually starting in childhood.
9. Juvenile macular degeneration: a rare inherited disorder that causes vision loss in young adults.
10. Retinal dystrophy: a group of inherited disorders that affect the retina and can cause progressive vision loss.

Retinal diseases can be diagnosed with a comprehensive eye exam, which includes a visual acuity test, dilated eye exam, and imaging tests such as optical coherence tomography (OCT) or fluorescein angiography. Treatment options vary depending on the specific disease and can include medication, laser surgery, or vitrectomy.

It's important to note that many retinal diseases can be inherited, so if you have a family history of eye problems, it's important to discuss your risk factors with your eye doctor. Early detection and treatment can help preserve vision and improve quality of life for those affected by these diseases.

While there is no cure for keratoconus, there are several treatment options available to help manage the condition. These include eyeglasses or contact lenses, specialized contact lenses called rigid gas permeable (RGP) lenses, and corneal transplantation in severe cases. Other treatments that may be recommended include phototherapeutic keratectomy (PTK), which involves removing damaged tissue from the cornea using a laser, or intacs, which are tiny plastic inserts that are placed into the cornea to flatten it and improve vision.

Keratoconus is relatively rare, affecting about 1 in every 2,000 people worldwide. However, it is more common in certain groups of people, such as those with a family history of the condition or those who have certain medical conditions, such as Down syndrome or sickle cell anemia. It typically affects both eyes, although one eye may be more severely affected than the other.

While there is no known cause for keratoconus, researchers believe that it may be linked to genetics, environmental factors, or a combination of both. The condition usually begins in adolescence or early adulthood and can progress over several years. In some cases, keratoconus can also be associated with other eye conditions, such as cataracts, glaucoma, or retinal detachment.

Epiretinal Membrane Treatment:

Surgical removal of the membrane is the most common treatment for epiretinal membrane. The procedure, called vitrectomy, involves removing the vitreous gel and the membrane from the eye. Laser photocoagulation can also be used to shrink the membrane and relieve symptoms.

It's important to note that not all epiretinal membranes require treatment. Some people may experience no vision problems and may not need any treatment at all. In other cases, the condition may resolve on its own over time. Your eye doctor will be able to determine the best course of action for your specific case.

Epiretinal Membrane Causes:

The exact cause of epiretinal membranes is not fully understood, but they are thought to arise from scar tissue that forms on the retina in response to injury or inflammation. They can also be associated with other eye conditions such as age-related macular degeneration, diabetic retinopathy, and retinal detachment.

Epiretinal Membrane Symptoms:

Symptoms of epiretinal membrane may include:

* Blurred vision
* Distorted vision (e.g., wavy lines or shapes)
* Difficulty reading or performing other daily tasks
* Metamorphopsia (visual distortion)

Epiretinal Membrane Diagnosis:

Your eye doctor will perform a comprehensive eye exam to diagnose epiretinal membrane. This may include a visual acuity test, dilated eye exam, and imaging tests such as optical coherence tomography (OCT) or ultrasonography.

Epiretinal Membrane Prognosis:

In some cases, epiretinal membranes can resolve on their own over time without treatment. However, if the membrane is causing significant vision loss or distortion, your eye doctor may recommend surgical removal. The prognosis for surgical treatment of epiretinal membrane is generally good, with many patients experiencing improved vision following the procedure.

Epiretinal Membrane Treatment:

Treatment for epiretinal membrane depends on the severity of symptoms and may include:

* Watchful waiting: In some cases, your eye doctor may recommend monitoring the membrane over time to see if it resolves on its own.
* Vitrectomy: This is a surgical procedure in which the vitreous gel is removed from the eye and the epiretinal membrane is removed or peeled off the retina.
* Laser photocoagulation: This is a non-surgical procedure that uses lasers to create small burns around the edges of the membrane, causing it to shrink and pull away from the retina.

Epiretinal Membrane Prevention:

There is no known way to prevent epiretinal membranes from forming, but there are some risk factors that may increase your likelihood of developing one. These include:

* Age: Epiretinal membranes are more common in older adults.
* Family history: If you have a family history of epiretinal membranes, you may be at higher risk.
* Previous eye surgery or trauma: People who have had eye surgery or suffered an eye injury may be at higher risk for developing an epiretinal membrane.

Epiretinal Membrane Prognosis:

The prognosis for epiretinal membranes is generally good, especially if the membrane is removed surgically. In some cases, vision may improve spontaneously over time without treatment. However, if left untreated, an epiretinal membrane can cause permanent vision loss. It is important to follow your eye doctor's recommendations for monitoring and treatment to ensure the best possible outcome.

Epiretinal Membrane Complications:

Complications of epiretinal membranes are rare but can include:

* Retinal detachment: This is a serious complication that occurs when the retina pulls away from the underlying tissue.
* Glaucoma: This is a condition that can cause vision loss and is often associated with increased pressure in the eye.
* Macular hole: This is a small hole in the macula, the part of the retina responsible for central vision.

Epiretinal Membrane Surgery:

If an epiretinal membrane is causing vision problems or is not improving with monitoring alone, surgery may be recommended. The goal of surgery is to remove the membrane and prevent it from recurring. There are several types of surgery that can be used to treat epiretinal membranes, including:

* Scleral buckle surgery: This involves sewing a flexible band around the eye to push the retina back into place.
* Vitrectomy: This is a procedure that removes the vitreous gel and any blood or scar tissue that may be causing problems.
* Photocoagulation: This is a procedure that uses laser light to shrink the membrane and seal off any leaky blood vessels.

It's important to note that not all epiretinal membranes require surgery, and in some cases, monitoring alone may be sufficient. Your eye doctor will be able to recommend the best course of treatment based on your specific condition and symptoms.

Symptoms of retinal perforations may include flashes of light, floaters, blurred vision, and loss of peripheral vision. These symptoms can be caused by a variety of factors, including age-related macular degeneration, diabetic retinopathy, and trauma to the eye.

Retinal perforations are typically diagnosed through a comprehensive eye exam, which may include imaging tests such as optical coherence tomography (OCT) and fluorescein angiography. Treatment for retinal perforations depends on the underlying cause of the condition, but may include laser surgery, cryotherapy, or vitrectomy.

In summary, retinal perforations are a serious condition that can cause significant vision loss if left untreated. Early detection and prompt treatment are essential to prevent long-term vision loss and improve outcomes for patients with retinal perforations.

There are several types of penetrating eye injuries, including:

1. Perforating injuries: These occur when an object punctures the globe of the eye, creating a hole in the retina or the sclera. These injuries can be life-threatening and require immediate medical attention.
2. Non-perforating injuries: These occur when an object does not penetrate the globe of the eye but still causes damage to the surrounding tissues. These injuries are typically less severe than perforating injuries but can still cause significant vision loss.
3. Hyphemas: These occur when blood collects in the space between the cornea and the iris, often due to a blow to the eye.
4. Retinal detachments: These occur when the retina becomes separated from the underlying tissue, often due to a traumatic injury.

Symptoms of penetrating eye injuries can include:

* Severe pain in the eye
* Redness and swelling of the affected eye
* Difficulty seeing or blindness
* Floaters or flashes of light
* A feeling of something in the eye

Treatment of penetrating eye injuries depends on the severity of the injury and can include:

1. Immediate medical attention to assess the extent of the injury and provide appropriate treatment.
2. Surgery to repair any damage to the eye, such as removing a foreign object or repairing a retinal detachment.
3. Antibiotics to prevent infection.
4. Pain management with medication.
5. Monitoring for complications, such as glaucoma or cataracts.

Preventive measures for penetrating eye injuries include:

1. Wearing protective eyewear when performing activities that could potentially cause eye injury, such as playing sports or working with power tools.
2. Avoiding touching the eyes or face to prevent the spread of infection.
3. Keeping the environment clean and free of hazards to reduce the risk of injury.
4. Properly storing and disposing of sharp objects to prevent accidents.
5. Seeking medical attention immediately if an eye injury occurs.

It is important to seek immediate medical attention if you experience any symptoms of a penetrating eye injury, as timely treatment can help prevent complications and improve outcomes.

The symptoms of RVO can vary depending on the severity of the blockage, but may include:

* Blurred vision
* Double vision
* Flashes of light
* Eye pain
* Reduced peripheral vision

RVO is typically diagnosed through a comprehensive eye exam, which may include imaging tests such as fluorescein angiography or optical coherence tomography (OCT).

Treatment for RVO depends on the severity of the condition and may include:

* Medications to reduce inflammation and improve blood flow
* Injections of medication into the eye
* Laser surgery to clear blockages or reduce inflammation
* Vitrectomy, a surgical procedure to remove the vitreous gel and blood from the eye

Early diagnosis and treatment of RVO can help prevent or reduce vision loss. However, in some cases, permanent vision loss may occur despite treatment.

Preventing RVO is not always possible, but controlling risk factors such as high blood pressure, diabetes, and hypertension can help reduce the likelihood of developing the condition. Maintaining a healthy lifestyle, including regular exercise, a balanced diet, and not smoking, can also help reduce the risk of RVO.

CNV develops when the underlying choroidal layers experience changes that lead to the growth of new blood vessels, which can leak fluid and cause damage to the retina. This can result in vision distortion, loss of central vision, and even blindness if left untreated.

The formation of CNV is a complex process that involves various cellular and molecular mechanisms. It is thought to be triggered by factors such as oxidative stress, inflammation, and the presence of certain growth factors and proteins.

There are several clinical signs and symptoms associated with CNV, including:

1. Distortion of vision, including metamorphopsia (distorted vision of geometric shapes)
2. Blind spots or scotomas
3. Decreased central vision
4. Difficulty reading or performing other daily tasks
5. Reduced color perception
6. Sensitivity to light and glare

The diagnosis of CNV is typically made based on a comprehensive eye exam, including a visual acuity test, dilated eye exam, and imaging tests such as fluorescein angiography or optical coherence tomography (OCT).

There are several treatment options for CNV, including:

1. Anti-vascular endothelial growth factor (VEGF) injections: These medications work by blocking the growth of new blood vessels and can help improve vision and reduce the risk of further damage.
2. Photodynamic therapy: This involves the use of a light-sensitive medication and low-intensity laser therapy to damage and shrink the abnormal blood vessels.
3. Focal photocoagulation: This involves the use of a high-intensity laser to destroy the abnormal blood vessels in the central retina.
4. Vitrectomy: In severe cases, a vitrectomy may be performed to remove the vitreous gel and blood vessels that are causing the CNV.

It is important to note that these treatments do not cure CNV, but they can help improve vision and slow the progression of the disease. Regular follow-up appointments with an eye care professional are necessary to monitor the condition and adjust treatment as needed.

There are several different types of congenital nystagmus, including:

1. Infantile nystagmus: This is the most common type and is present in approximately 10% of infants. It is often associated with other conditions such as hypertrophy of the retina or abnormalities of the optic nerve.
2. Accommodative nystagmus: This type is caused by a problem with the eye's ability to focus and can be treated with glasses or contact lenses.
3. Ocular motor nystagmus: This type is caused by a problem with the eye muscles and can be associated with conditions such as cerebral palsy or down syndrome.
4. Optokinetic nystagmus: This type is caused by a problem with the visual pathway and can be associated with conditions such as stroke or traumatic brain injury.

Congenital nystagmus can be diagnosed through a comprehensive eye exam, which may include a visual acuity test, refraction test, and an assessment of eye movements using a technique called electronystagmography (ENG). Treatment for congenital nystagmus depends on the underlying cause and may include glasses or contact lenses, prism lenses, or in some cases, surgery.

It is important to note that congenital nystagmus can have a significant impact on an individual's vision and quality of life, and it is important to seek medical attention if symptoms persist or worsen over time.

Pathological nystagmus can be diagnosed through a comprehensive eye examination, including a visual acuity test, refraction test, cover test, and eyer movements assessment. Imaging studies such as CT or MRI scans may also be ordered to rule out other possible causes of the symptoms.

Treatment for pathological nystagmus depends on the underlying cause of the condition. In some cases, treatment may involve correcting refractive errors or addressing any underlying brain disorders through medication, physical therapy, or surgery. Other treatments may include eye exercises, prisms, or specialized glasses to help improve eye movement and reduce the symptoms of nystagmus.

In summary, pathological nystagmus is an abnormal and involuntary movement of the eyeballs that can be caused by various neurological disorders. Diagnosis is through a comprehensive eye examination and imaging studies, and treatment depends on the underlying cause of the condition.

1. Keratoconus: This is a progressive thinning of the cornea that can cause it to bulge into a cone-like shape, leading to blurred vision and sensitivity to light.
2. Fuchs' dystrophy: This is a condition in which the cells in the innermost layer of the cornea become damaged, leading to clouding and blurred vision.
3. Bullous keratopathy: This is a condition in which there is a large, fluid-filled bubble on the surface of the cornea, which can cause blurred vision and discomfort.
4. Corneal ulcers: These are open sores on the surface of the cornea that can be caused by infection or other conditions.
5. Dry eye syndrome: This is a condition in which the eyes do not produce enough tears, leading to dryness, irritation, and blurred vision.
6. Corneal abrasions: These are scratches on the surface of the cornea that can be caused by injury or other conditions.
7. Trachoma: This is an infectious eye disease that can cause scarring and blindness if left untreated.
8. Ocular herpes: This is a viral infection that can cause blisters on the surface of the cornea and lead to scarring and vision loss if left untreated.
9. Endophthalmitis: This is an inflammation of the inner layer of the eye that can be caused by bacterial or fungal infections, and can lead to severe vision loss if left untreated.
10. Corneal neovascularization: This is the growth of new blood vessels into the cornea, which can be a complication of other conditions such as dry eye syndrome or ocular trauma.

These are just a few examples of the many different types of corneal diseases that can affect the eyes. It's important to seek medical attention if you experience any symptoms such as pain, redness, or blurred vision in one or both eyes. Early diagnosis and treatment can help prevent complications and preserve vision.

Scotoma is a term that was first used in the early 19th century to describe blind spots in the visual field caused by defects in the retina or optic nerve. Over time, the term has been broadened to include any type of blind spot or defect in the visual field, regardless of its cause.

There are several different types of scotomas, including:

1. Homonymous hemianopsia: A condition in which there is a blind spot in one side of both eyes, causing difficulty with recognizing objects and people on that side.
2. Hemianopia: A condition in which there is a blind spot in one half of both eyes, often caused by a stroke or brain injury.
3. Quadrantanopia: A condition in which there is a blind spot in one quarter of both eyes, often caused by a stroke or brain injury.
4. Scanning vision: A condition in which the visual field appears to be scanned or sectioned off, often caused by a brain disorder such as multiple sclerosis.
5. Blind spot scotoma: A condition in which there is a small blind spot in the central part of the visual field, often caused by a lesion in the retina or optic nerve.

Scotomas can have a significant impact on daily life, making it difficult to perform everyday tasks such as driving, reading, and recognizing faces. Treatment options for scotomas depend on the underlying cause and may include prism glasses, vision therapy, or surgery. In some cases, scotomas may be a sign of a more serious condition that requires medical attention.

There are several types of color vision defects, including:

1. Color blindness: This is a common condition where individuals have difficulty distinguishing between certain colors, such as red and green. It is usually inherited and affects males more frequently than females.
2. Achromatopsia: This is a rare condition where individuals have difficulty seeing any colors and only see shades of gray.
3. Tritanopia: This is a rare condition where individuals have difficulty seeing the color blue and only see yellow and red.
4. Deuteranomaly: This is a common condition where individuals have difficulty seeing red and green colors and see these colors as more yellow or orange.
5. Anomalous trichromacy: This is a rare condition where individuals have an extra type of cone in their retina, which can cause unusual color perception.

Color vision defects can be diagnosed with a series of tests, including the Ishihara test, the Farnsworth-Munsell 100 Hue Test, and the Lantern Test. Treatment options vary depending on the type and severity of the condition, but may include glasses or contact lenses, color filters, or surgery.

In conclusion, color vision defects can significantly impact daily life, making it important to be aware of these conditions and seek medical attention if symptoms persist or worsen over time. With proper diagnosis and treatment, individuals with color vision defects can lead normal and fulfilling lives.

The symptoms of RP can vary depending on the severity of the condition and the specific genetic mutations causing it. Common symptoms include:

* Night blindness
* Difficulty seeing in low light environments
* Blind spots or missing areas in central vision
* Difficulty reading or recognizing faces
* Sensitivity to light
* Reduced peripheral vision
* Blurred vision

There is currently no cure for RP, and treatment options are limited. However, researchers are actively working to develop new therapies and technologies to slow the progression of the disease and improve the quality of life for individuals with RP. These include:

* Gene therapy: Using viral vectors to deliver healthy copies of the missing gene to the retina in an effort to restore normal vision.

* Stem cell therapy: Transplanting healthy stem cells into the retina to replace damaged or missing cells.

* Pharmacological interventions: Developing drugs that can slow down or reverse the progression of RP by targeting specific molecular pathways.

* Retinal implants: Implanting a retinal implant, such as a retinal prosthetic, to bypass damaged or non-functional photoreceptors and directly stimulate the visual pathway.

It's important to note that these therapies are still in the experimental stage and have not yet been proven effective in humans. Therefore, individuals with RP should consult with their healthcare provider about the best treatment options available.

In summary, Retinitis Pigmentosa is a genetic disorder that causes progressive vision loss, particularly during childhood or adolescence. While there is currently no cure for RP, researchers are actively working to develop new therapies to slow down or restore vision in those affected by the disease. These include gene therapy, stem cell therapy, pharmacological interventions, and retinal implants. It's important to consult with a healthcare provider for the best treatment options available.

FAQs:

1. What is Retinitis Pigmentosa?

Retinitis Pigmentosa (RP) is a genetic disorder that causes progressive vision loss, typically during childhood or adolescence.

2. What are the symptoms of Retinitis Pigmentosa?

Symptoms of RP can vary depending on the specific mutation causing the disease, but common symptoms include difficulty seeing at night, loss of peripheral vision, and difficulty adjusting to bright light.

3. Is there a cure for Retinitis Pigmentosa?

Currently, there is no cure for RP, but researchers are actively working on developing new therapies to slow down or restore vision in those affected by the disease.

4. What are some potential treatments for Retinitis Pigmentosa?

Some potential treatments for RP include gene therapy, stem cell therapy, pharmacological interventions, and retinal implants. It's important to consult with a healthcare provider for the best treatment options available.

5. Can Retinitis Pigmentosa be prevented?

RP is a genetic disorder, so it cannot be prevented in the classical sense. However, researchers are working on developing gene therapies that can prevent or slow down the progression of the disease.

6. How does Retinitis Pigmentosa affect daily life?

Living with RP can significantly impact daily life, especially as vision loss progresses. It's important to adapt and modify daily routines, such as using assistive devices like canes or guide dogs, and seeking support from family and friends.

7. What resources are available for those affected by Retinitis Pigmentosa?

There are a variety of resources available for those affected by RP, including support groups, advocacy organizations, and online communities. These resources can provide valuable information, support, and connections with others who understand the challenges of living with the disease.

There are several different types of glaucoma, including:

* Open-angle glaucoma: This is the most common form of glaucoma, and is caused by slowed drainage of fluid from the eye.
* Closed-angle glaucoma: This type of glaucoma is caused by a blockage in the drainage channels of the eye, leading to a sudden increase in pressure.
* Normal-tension glaucoma: This type of glaucoma is caused by damage to the optic nerve even though the pressure in the eye is within the normal range.
* Congenital glaucoma: This is a rare type of glaucoma that is present at birth, and is caused by a developmental defect in the eye's drainage system.

Symptoms of glaucoma can include:

* Blurred vision
* Loss of peripheral vision
* Eye pain or pressure
* Redness of the eye
* Seeing halos around lights

Glaucoma is typically diagnosed with a combination of visual acuity tests, dilated eye exams, and imaging tests such as ultrasound or MRI. Treatment for glaucoma usually involves medication to reduce pressure in the eye, but may also include surgery to improve drainage or laser therapy to prevent further damage to the optic nerve.

Early detection and treatment of glaucoma is important to prevent vision loss, so it is important to have regular eye exams, especially if you are at risk for the condition. Risk factors for glaucoma include:

* Age (over 60)
* Family history of glaucoma
* Diabetes
* High blood pressure
* African or Hispanic ancestry

Overall, glaucoma is a serious eye condition that can cause vision loss if left untreated. Early detection and treatment are key to preventing vision loss and maintaining good eye health.

A vitreous hemorrhage is a type of eye injury that occurs when there is bleeding within the vitreous humor, the gel-like substance that fills the space between the lens and the retina of the eye. This condition can be caused by a variety of factors, such as trauma, disease, or complications during surgery.

Symptoms of Vitreous Hemorrhage[2]

The symptoms of vitreous hemorrhage can vary depending on the severity of the injury, but may include:

* Blurred vision
* Floaters (specks or cobwebs in vision)
* Flashes of light
* Eye pain
* Redness and swelling of the eye
* Sensitivity to light

Treatment of Vitreous Hemorrhage[2]

The treatment of vitreous hemorrhage depends on the underlying cause and severity of the injury. In some cases, the body may absorb the blood over time, and no treatment is necessary. However, if the hemorrhage is severe or causing significant vision loss, surgery may be required to remove the blood and repair any damage to the eye.

Complications of Vitreous Hemorrhage[2]

If left untreated, vitreous hemorrhage can lead to a number of complications, including:

* Glaucoma (increased pressure in the eye)
* Retinal detachment (separation of the retina from the back of the eye)
* Cataract formation
* Infection
* Blindness

Prevention of Vitreous Hemorrhage[2]

While some cases of vitreous hemorrhage cannot be prevented, there are steps that can be taken to reduce the risk of developing this condition. These include:

* Wearing protective eyewear during activities that could potentially cause eye injury
* Avoiding close-range objects or activities that could cause trauma to the eye
* Getting regular eye exams to monitor for any changes or abnormalities in the eye

In conclusion, vitreous hemorrhage is a serious condition that can cause significant vision loss if left untreated. While some cases may resolve on their own over time, others may require surgical intervention to prevent complications and preserve vision. Regular eye exams and protective eyewear can help reduce the risk of developing this condition.

References:

[1] American Academy of Ophthalmology. (2020). Vitreous Hemorrhage. Retrieved from

[2] MedlinePlus. (2020). Vitreous Hemorrhage. Retrieved from

Hyperopia, also known as farsightedness, is a common vision condition in which close objects appear blurry while distant objects appear clear. This occurs when the eyeball is shorter than normal or the cornea is not curved enough, causing light rays to focus behind the retina rather than directly on it. Hyperopia can be treated with glasses, contact lenses, or refractive surgery.

Word origin: Greek "hyper" (beyond) + "ops" (eye) + -ia (suffix denoting a condition or state)

First recorded use: 1690s

The term "presbyopia" comes from the Greek words "presbys," meaning "old," and "ops," meaning "eye." It was first described by the English physician and surgeon Thomas Wharton in 1655, and has since become a widely recognized condition in the field of ophthalmology.

Presbyopia is caused by a loss of elasticity in the lens of the eye, which makes it less able to change shape and focus on close objects. This can be exacerbated by other age-related changes such as cataracts, glaucoma, or macular degeneration.

Symptoms of presbyopia include difficulty reading or performing other close-up tasks, headaches or eye strain from trying to focus, and blurred vision. Treatment options for presbyopia include corrective lenses such as glasses or contact lenses, bifocal or multifocal lenses, or surgical procedures such as refractive surgery or intraocular lens implantation.

Overall, presbyopia is a common and treatable condition that can significantly impact an individual's quality of life, particularly as they age. It is important for individuals to have regular eye exams to monitor their vision and address any changes in their eyesight over time.

Endophthalmitis can be classified into several types based on its causes, such as:

1. Postoperative endophthalmitis: This type of endophthalmitis occurs after cataract surgery or other intraocular surgeries. It is caused by bacterial infection that enters the eye through the surgical incision.
2. Endogenous endophthalmitis: This type of endophthalmitis is caused by an infection that originates within the eye, such as from a retinal detachment or uveitis.
3. Exogenous endophthalmitis: This type of endophthalmitis is caused by an infection that enters the eye from outside, such as from a penetrating injury or a foreign object in the eye.

The symptoms of endophthalmitis can include:

1. Severe pain in the eye
2. Redness and swelling of the conjunctiva
3. Difficulty seeing or blind spots in the visual field
4. Sensitivity to light
5. Increased sensitivity to touch or pressure on the eye
6. Fever and chills
7. Swollen lymph nodes
8. Enlarged pupil
9. Clouding of the vitreous humor

If you suspect that you or someone else has endophthalmitis, it is important to seek medical attention immediately. Early diagnosis and treatment can help prevent vision loss. Treatment options for endophthalmitis may include antibiotics, vitrectomy (removal of the vitreous humor), and in some cases, removal of the affected eye.

Pseudophakia is considered a rare condition, as most cataract surgeries involve removal of the entire natural lens. However, there are certain situations where leaving behind some residual lens material can be beneficial, such as in cases where the patient has severe astigmatism or presbyopia (age-related loss of near vision).

The presence of pseudophakia can affect the visual outcome and refractive status of the eye, and may require additional surgical intervention to optimize visual acuity. It is important for ophthalmologists to be aware of this condition and consider it when evaluating patients with cataracts or other eye conditions.

Optic atrophy is a condition where there is a degeneration or loss of the optic nerve fibers, leading to vision loss. It can be caused by various factors such as trauma, inflammation, tumors, and certain medical conditions like multiple sclerosis.

The symptoms of optic atrophy may include:

1. Blind spots in the visual field
2. Difficulty perceiving colors
3. Difficulty adjusting to bright light
4. Double vision or other abnormalities in binocular vision
5. Eye pain or discomfort
6. Loss of peripheral vision
7. Nausea and vomiting
8. Sensitivity to light
9. Tunnel vision
10. Weakness or numbness in the face or extremities.

The diagnosis of optic atrophy is based on a comprehensive eye exam, which includes a visual acuity test, dilated eye exam, and other specialized tests such as an OCT (optical coherence tomography) scan.

Treatment for optic atrophy depends on the underlying cause and may include medications to manage inflammation or infection, surgery to remove a tumor or repair damaged tissue, or management of associated conditions such as diabetes or multiple sclerosis. In some cases, vision loss due to optic atrophy may be permanent and cannot be reversed, but there are strategies to help improve remaining vision and adapt to any visual impairment.

Retinal hemorrhage can cause vision loss or blindness if not treated promptly. The bleeding can lead to scarring, which can cause permanent damage to the retina and affect vision. In some cases, retinal hemorrhage can be a sign of a more serious underlying condition that requires immediate medical attention.

Retinal hemorrhage is diagnosed through a comprehensive eye exam, which includes a visual acuity test, dilated eye exam, and imaging tests such as fluorescein angiography or optical coherence tomography. Treatment options for retinal hemorrhage depend on the underlying cause and can include laser surgery, medication, or vitrectomy.

In summary, retinal hemorrhage is a serious condition that can cause vision loss or blindness if not treated promptly. It is essential to seek medical attention if symptoms such as blurred vision, flashes of light, or floaters are noticed. Early detection and treatment can help prevent or reduce vision loss in cases of retinal hemorrhage.

The test works by shining a light into the eye and measuring the way the light is distorted as it passes through the cornea. This distortion is caused by the curvature of the cornea and by any imperfections or abnormalities in its surface. The resulting distortion is called a "wavefront aberration."

The CWA test produces a map of the wavefront aberrations in the eye, which can be used to identify specific conditions and to determine the appropriate treatment. The test is painless and takes only a few minutes to perform.

CWA is commonly used to diagnose and monitor a range of eye conditions, including:

1. Astigmatism: This is a condition in which the cornea is irregularly shaped, causing blurred vision at all distances.
2. Nearsightedness (myopia): This is a condition in which close objects are seen clearly, but distant objects appear blurry.
3. Farsightedness (hyperopia): This is a condition in which distant objects are seen clearly, but close objects appear blurry.
4. Keratoconus: This is a progressive thinning of the cornea that can cause distorted vision and increase the risk of complications such as corneal scarring or blindness.
5. Other conditions such as presbyopia (age-related loss of near vision), amblyopia (lazy eye), and ocular injuries.

Overall, CWA is a valuable diagnostic tool for assessing the quality of the cornea and for diagnosing and monitoring a range of eye conditions. It can help eye care professionals to identify the underlying causes of vision problems and to develop effective treatment plans to improve vision and prevent complications.

Wet macular degeneration can cause vision loss in several ways:

1. Leakage of fluid from the new blood vessels can cause swelling and distortion of the retina, leading to vision loss.
2. The growth of new blood vessels can cause scar tissue to form, which can pull on the retina and cause it to tear or detach, leading to vision loss.
3. The buildup of fluid and scar tissue can cause the macula, the part of the retina responsible for central vision, to become distorted or atrophic, leading to vision loss.

Symptoms of wet macular degeneration may include:

1. Blurred vision
2. Distorted vision (e.g., straight lines appear wavy)
3. Difficulty reading or recognizing faces
4. Difficulty adjusting to bright light
5. Floaters (specks or cobwebs in your vision)
6. Blind spots in your central vision

If you experience any of these symptoms, it is important to see an eye doctor as soon as possible for a comprehensive evaluation and treatment. Wet macular degeneration can be treated with anti-vascular endothelial growth factor (VEGF) injections, photodynamic therapy, or a combination of both. These treatments aim to reduce the growth of new blood vessels and slow down the progression of the disease.

It is important to note that wet macular degeneration can progress rapidly, so early detection and treatment are crucial to preserving vision. Regular eye exams, especially after age 50, can help detect wet macular degeneration in its early stages, when it is more treatable.

There are several different types of uveitis, including:

1. Anterior uveitis: This type affects the front part of the eye and is the most common form of uveitis. It is often caused by an infection or injury.
2. Posterior uveitis: This type affects the back part of the eye and can be caused by a systemic disease such as sarcoidosis or juvenile idiopathic arthritis.
3. Intermediate uveitis: This type affects the middle layer of the eye and is often caused by an autoimmune disorder.
4. Panuveitis: This type affects the entire uvea and can be caused by a systemic disease such as vasculitis or Behçet's disease.

Symptoms of uveitis may include:

* Eye pain
* Redness and swelling in the eye
* Blurred vision
* Sensitivity to light
* Floaters (specks or cobwebs in your vision)
* Flashes of light

If you experience any of these symptoms, it is important to see an eye doctor as soon as possible. Uveitis can be diagnosed with a comprehensive eye exam, which may include imaging tests such as ultrasound or MRI. Treatment for uveitis depends on the cause and severity of the condition, but may include medication to reduce inflammation, antibiotics for infections, or surgery to remove any diseased tissue.

Early diagnosis and treatment are important to prevent complications such as cataracts, glaucoma, and blindness. If you have uveitis, it is important to follow your doctor's recommendations for treatment and monitoring to protect your vision.

Some common examples of choroid diseases include:

1. Choroidal neovascularization (CNV): This is a condition where new blood vessels grow under the retina, often as a result of age-related macular degeneration (AMD) or other eye conditions. These new vessels can cause vision loss and distortion.
2. Choroidal melanoma: This is a type of cancer that develops in the choroid layer of the eye. It is usually slow-growing, but it can spread to other parts of the body if left untreated.
3. Choroiditis: This is an inflammatory condition that affects the choroid layer of the eye, often as a result of infection or autoimmune disorders. It can cause vision loss and pain in the affected eye.
4. Choroidal rupture: This is a rare condition where the choroid layer of the eye ruptures, leading to bleeding and potentially severe vision loss.
5. Other conditions: There are several other conditions that can affect the choroid layer of the eye, such as choroidal vasculitis, choroidal effusion, and choroidal tumors. These conditions can cause a range of symptoms, including vision loss, pain, and distortion.

Overall, choroid diseases can have a significant impact on vision and eye health, and it is important to seek medical attention if any symptoms persist or worsen over time. Early detection and treatment can help to mitigate the risk of long-term vision loss and other complications.

The symptoms of optic neuritis may include:

* Blurred vision or loss of vision
* Eye pain or pressure
* Sensitivity to light
* Dimness of colors
* Difficulty moving the eyes
* Numbness or weakness in the face

The cause of optic neuritis is not always known, but it is believed to be related to an abnormal immune response. In MS, optic neuritis is thought to be triggered by the immune system attacking the protective covering of nerve fibers in the central nervous system.

Treatment for optic neuritis depends on the underlying cause. In cases of MS, treatment with corticosteroids can help reduce inflammation and slow the progression of the disease. In other conditions, treatment may involve addressing the underlying cause, such as an infection or a tumor.

Prognosis for optic neuritis varies depending on the underlying cause. In MS, the condition can recur and lead to long-term vision loss if left untreated. However, with prompt treatment and management, many people with MS experience significant improvement in their vision.

There are two main types of retinal artery occlusion: central retinal artery occlusion (CRAO) and branch retinal artery occlusion (BRAO). Central retinal artery occlusion occurs when the central retinal artery, which supplies blood to the macula, becomes blocked. This can cause sudden vision loss in one eye, often with a painless, blinding effect. Branch retinal artery occlusion, on the other hand, occurs when one of the smaller retinal arteries that branch off from the central retinal artery becomes blocked. This can cause vision loss in a specific part of the visual field, often with some preserved peripheral vision.

Retinal artery occlusion is often caused by a blood clot or other debris that blocks the flow of blood through the retinal arteries. It can also be caused by other conditions such as diabetes, high blood pressure, and atherosclerosis (the buildup of plaque in the arteries).

Retinal artery occlusion is a medical emergency that requires prompt treatment. Treatment options may include intravenous injection of medications to dissolve the clot or other debris, laser surgery to repair damaged retinal tissue, and/or vitrectomy (surgical removal of the vitreous gel) to remove any blood or debris that has accumulated in the eye.

In summary, retinal artery occlusion is a serious condition that can cause sudden vision loss and potentially lead to permanent blindness. It is important to seek medical attention immediately if you experience any symptoms of retinal artery occlusion, such as sudden vision loss or blurred vision in one eye, flashes of light, floaters, or pain in the eye.

The exact cause of CSR is not fully understood, but it is thought to be related to changes in blood flow and inflammation in the retina. It can occur in people of all ages and is more common in males than females.

Symptoms of CSR may include:

* Blurred vision or blind spots
* Distorted vision
* Sensitivity to light
* Floating objects or cobwebs in vision
* Eye pain or discomfort

Diagnosis of CSR typically involves a comprehensive eye exam, including a visual acuity test, dilated eye exam, and imaging tests such as optical coherence tomography (OCT).

Treatment for CSR depends on the underlying cause and severity of the condition. In some cases, no treatment may be necessary, as the condition may resolve on its own over time. Other treatments may include:

* Medications to reduce inflammation and improve blood flow in the retina
* Photodynamic therapy (PDT) to reduce the amount of fluid in the retina
* Vitrectomy, a surgical procedure to remove the vitreous gel and relieve pressure on the retina.

It is important for individuals with CSR to follow their treatment plan and have regular follow-up appointments with their eye care professional to monitor the progression of the condition and adjust treatment as needed. With appropriate treatment, many people with CSR experience improvement in their vision.

There are several types of fungal eye infections, including:

1. Aspergillosis: This is a common type of fungal infection that affects the eye. It is caused by the fungus Aspergillus and can occur in people with weakened immune systems or pre-existing eye conditions.
2. Candidemia: This is another common type of fungal infection that affects the eye. It is caused by the fungus Candida and can occur in people with weakened immune systems or pre-existing eye conditions.
3. Cryptococcosis: This is a rare type of fungal infection that affects the eye. It is caused by the fungus Cryptococcus and can occur in people with weakened immune systems, such as those with HIV/AIDS.
4. Histoplasmosis: This is a rare type of fungal infection that affects the eye. It is caused by the fungus Histoplasma and can occur in people who have been exposed to the fungus in soil or bird droppings.
5. Blastomycosis: This is a rare type of fungal infection that affects the eye. It is caused by the fungus Blastomyces and can occur in people who have been exposed to the fungus in soil or water.

Fungal eye infections can cause a range of symptoms, including redness, discharge, pain, and vision loss. Treatment typically involves antifungal medication and may also include surgery to remove any infected tissue. In severe cases, fungal eye infections can lead to blindness if left untreated.

Prevention measures for fungal eye infections include good hygiene practices, such as washing hands regularly and avoiding close contact with people who have the infection. People with weakened immune systems should also avoid exposure to fungi by avoiding outdoor activities during peak fungal growth seasons and wearing protective clothing when working or playing in areas where fungi are likely to be present.

Overall, fungal eye infections are uncommon but can be serious conditions that require prompt medical attention. If you suspect you may have a fungal eye infection, it is important to seek medical care as soon as possible to receive proper diagnosis and treatment.

Symptoms of uveitis, intermediate may include:

* Blurred vision
* Eye pain
* Redness and swelling of the eye
* Sensitivity to light
* Floaters (specks or cobwebs in vision)

Uveitis, intermediate can be caused by a variety of factors, including infection, injury, or autoimmune disorders. It is important to seek medical attention if symptoms persist or worsen over time, as early diagnosis and treatment can help prevent long-term damage to the eye.

Treatment options for uveitis, intermediate may include:

* Corticosteroids to reduce inflammation
* Anti-inflammatory medications
* Immunosuppressive drugs to suppress the immune system and prevent further inflammation
* Surgery to remove inflamed tissue or repair damaged areas of the eye.

Prognosis for uveitis, intermediate depends on the severity of the disease and the promptness and effectiveness of treatment. In general, early diagnosis and treatment can lead to a good prognosis with minimal long-term complications. However, if left untreated or if the disease is severe, it can lead to permanent vision loss and other complications such as cataracts, glaucoma, and eye pain.

Uveitis, posterior refers to inflammation affecting the uvea, specifically the choroid and retina at the rear of the eye. This type of uveitis is characterized by symptoms such as blurred vision, sensitivity to light, floaters, and redness in the affected eye.

Etymology of Uveitis, Posterior

The word "uveitis" comes from the Latin words "uva," meaning grape, and "itis," meaning inflammation. This refers to the fact that the uvea is structured like a layer of grapes, with each layer containing blood vessels and connective tissue. The suffix "posterior" indicates that the inflammation affects the rear portion of the eye.

Prevalence of Uveitis, Posterior

Uveitis, posterior is relatively uncommon, accounting for approximately 5% to 10% of all uveitis cases. However, it is a leading cause of legal blindness in working-age adults. The prevalence varies by population and geographic location, with higher rates found in certain ethnic groups such as Hispanics and Asians.

Risk Factors for Uveitis, Posterior

Several factors increase the risk of developing uveitis, posterior, including:

1. Age: The condition primarily affects adults between 20 and 50 years old.
2. Gender: Women are more likely to be affected than men.
3. Ethnicity: Certain ethnic groups such as Hispanics and Asians have a higher risk of developing uveitis, posterior.
4. Family history: Individuals with a family history of uveitis or other autoimmune diseases are more likely to develop the condition.
5. Previous ocular surgery: People who have undergone cataract or vitrectomy surgery may be at higher risk for developing posterior uveitis.
6. Systemic conditions: Certain medical conditions such as rheumatoid arthritis, multiple sclerosis, and sarcoidosis increase the risk of developing uveitis, posterior.
7. Medications: Certain medications such as corticosteroids and immunosuppressive drugs may increase the risk of developing uveitis, posterior.

Symptoms of Uveitis, Posterior

The symptoms of uveitis, posterior can vary depending on the severity of the inflammation and the location of the affected tissue. Common symptoms include:

1. Blurred vision
2. Eye pain or discomfort
3. Sensitivity to light
4. Redness and inflammation in the eye
5. Floaters or flashes of light
6. Blind spots or missing areas of vision
7. Cloudy or hazy vision

Diagnosis of Uveitis, Posterior

The diagnosis of uveitis, posterior is based on a combination of clinical evaluation and diagnostic tests. A comprehensive ophthalmological examination includes:

1. Visual acuity testing
2. Dilated eye exam
3. Intraocular pressure measurement
4. Funduscopic examination
5. Optical coherence tomography (OCT)
6. Fluorescein angiography
7. Laboratory tests, such as blood tests or lumbar puncture, to rule out systemic diseases.

Treatment of Uveitis, Posterior

The treatment of uveitis, posterior depends on the severity and location of the inflammation, as well as the underlying cause. Treatment options may include:

1. Topical or systemic corticosteroids to reduce inflammation
2. Immunosuppressive drugs to suppress the immune response
3. Antiviral or antibacterial medications to treat infections
4. Laser therapy to reduce inflammation and prevent further vision loss
5. Vitrectomy, a surgical procedure to remove the vitreous gel and inflammatory cells.

Prognosis of Uveitis, Posterior

The prognosis of uveitis, posterior is generally good if treated promptly and effectively. The majority of patients with this condition can expect to regain their vision and avoid long-term complications. However, in some cases, the inflammation can cause permanent damage to the retina and optic nerve, leading to vision loss or even blindness.

Complications of Uveitis, Posterior

Uveitis, posterior can lead to several complications, including:

1. Cataract formation
2. Glaucoma
3. Retinal detachment
4. Vitreous hemorrhage
5. Macular edema
6. Optic nerve damage
7. Increased intracranial pressure
8. Meningitis or encephalitis

Prevention of Uveitis, Posterior

There is no definitive way to prevent uveitis, posterior, but early detection and prompt treatment can help reduce the risk of complications. Good eye care practices, such as regular eye exams and proper management of systemic diseases, can also help prevent or manage the condition.

In conclusion, uveitis, posterior is a serious condition that can cause significant vision loss if left untreated. Early detection and prompt treatment are crucial to prevent long-term complications and preserve vision. A comprehensive eye exam and proper management of systemic diseases can help prevent or manage the condition.

The symptoms of a corneal ulcer may include:

* Pain or discomfort in the eye
* Redness and swelling of the eye
* Discharge or pus in the eye
* Blurred vision or sensitivity to light
* A feeling that there is something in the eye

If left untreated, a corneal ulcer can lead to complications such as:

* Perforation of the cornea
* Inflammation of the iris (iritis)
* Inflammation of the retina (retinitis)
* Vision loss or blindness

Treatment of a corneal ulcer typically involves antibiotic eye drops or ointments to treat any underlying bacterial infection, as well as supportive care to manage pain and promote healing. In severe cases, surgery may be necessary to remove the damaged tissue and promote healing.

Prevention of corneal ulcers includes good hygiene, proper use of contact lenses, and avoiding touching or rubbing the eyes. Early detection and treatment are key to preventing complications and preserving vision.

Types of Eye Foreign Bodies:

There are several types of eye foreign bodies, including:

1. Dust and small particles: These are the most common type of eye foreign body and can enter the eye through contact with the environment or by rubbing the eyes.
2. Large objects: These can include splinters, pen tips, or other small objects that become lodged in the eye.
3. Chemical irritants: Chemicals like household cleaners or pesticides can irritate the eyes and cause foreign body sensation.
4. Microorganisms: Bacteria, viruses, and other microorganisms can enter the eye and cause inflammation, which can lead to a foreign body sensation.

Symptoms of Eye Foreign Bodies:

The symptoms of an eye foreign body can vary depending on the size and location of the object, but common signs include:

1. Redness and irritation
2. Itching or burning sensation in the eye
3. Discharge or tearing
4. Blurred vision or sensitivity to light
5. Pain or discomfort in the eye

Diagnosis and Treatment of Eye Foreign Bodies:

If you suspect that you have an eye foreign body, it is important to seek medical attention as soon as possible. A healthcare professional will perform a thorough examination of the eye to locate the foreign body and determine the best course of treatment.

Treatment for eye foreign bodies may include:

1. Flushing the eye with water or saline solution to try to dislodge the object
2. Using antibiotic drops or ointments to prevent infection
3. Removing the object with a special instrument, such as a cotton swab or forceps
4. In severe cases, surgery may be necessary to remove the foreign body.

Prevention of Eye Foreign Bodies:

To prevent eye foreign bodies, it is important to take the following precautions:

1. Avoid touching or rubbing your eyes, as this can introduce bacteria and other contaminants into the eye.
2. Keep your hands and face clean, especially after handling chemicals or other potentially harmful substances.
3. Wear protective eyewear, such as goggles or safety glasses, when working with power tools or other equipment that can generate debris.
4. Avoid wearing contact lenses while swimming or in other wet environments.
5. Keep your home and work environment clean and free of clutter to reduce the risk of objects becoming lodged in the eye.

Conclusion:

Eye foreign bodies can cause a range of symptoms, from mild discomfort to serious vision loss. If you suspect that you have an object stuck in your eye, it is important to seek medical attention as soon as possible. With prompt diagnosis and appropriate treatment, most eye foreign bodies can be successfully removed and the risk of complications minimized. By taking precautions to prevent eye injuries and seeking immediate medical care if you experience any symptoms, you can help protect your vision and maintain good eye health.

Benign optic nerve neoplasms, such as meningiomas and melanocytic nevi, are relatively common and may not require treatment unless they become large enough to compress the optic nerve or cause other complications. Malignant optic nerve neoplasms, such as retinoblastoma and lung metastases, are less common but can be more aggressive and require prompt treatment to prevent further damage.

Symptoms of optic nerve neoplasms can include blurred vision, double vision, eye pain, and loss of peripheral vision. Diagnosis is typically made through a combination of imaging tests such as MRI or CT scans, and visual field testing to assess the extent of the tumor and its effects on the optic nerve.

Treatment options for optic nerve neoplasms depend on the type and location of the tumor, as well as the severity of any symptoms. Benign tumors may be monitored with regular imaging studies to ensure that they do not grow or become more aggressive, while malignant tumors may require surgery, chemotherapy, or radiation therapy to remove the tumor and prevent further damage. In some cases, treatment may involve a combination of these approaches.

Overall, optic nerve neoplasms are rare but potentially serious conditions that can affect vision and eye health. Early diagnosis and treatment are important to help preserve vision and prevent complications.

Example Sentences:

1. The patient was diagnosed with iris disease and was prescribed antibiotic eye drops to help clear up the infection.
2. The doctor suspected that the patient's blurred vision was caused by an iris disease, so he referred the patient to a specialist for further evaluation.
3. Although the symptoms of iris disease can be uncomfortable, most cases can be effectively treated with medication and proper care.

Lens subluxation is a condition where the crystalline lens of the eye partially or completely dislocates from its normal position behind the iris, causing visual impairment and potential vision loss. It can occur due to various causes such as trauma, inflammation, or degenerative changes.

The term "subluxation" means a partial dislocation of a structure, and in the context of the crystalline lens, it refers to the displacement of the lens from its normal position in the eye.

Lens subluxation can be classified into several types based on the location and extent of the displacement:

1. Posterior subluxation: The lens is displaced backward, away from the iris.
2. Anterior subluxation: The lens is displaced forward, towards the iris.
3. Total subluxation: The lens is completely dislocated from its normal position.

Symptoms of lens subluxation can include blurred vision, double vision, eye pain, and sensitivity to light. Treatment options depend on the severity and cause of the condition, and may include glasses or contact lenses, medication, or surgery.

In summary, lens subluxation is a condition where the crystalline lens of the eye partially or completely dislocates from its normal position, leading to visual impairment and potential vision loss. It can occur due to various causes and can be classified into several types based on the location and extent of the displacement.

The term "papilledema" comes from the Greek words "papilla," meaning "little nipple," and "dema," meaning "swelling." This refers to the appearance of the optic disc when it is swollen, as it looks like a small, round nipple on the surface of the retina.

Papilledema can be caused by a variety of conditions, including high blood pressure, brain tumors, and aneurysms. It can also be a symptom of other conditions such as meningitis or multiple sclerosis. The diagnosis of papilledema is typically made through a comprehensive eye exam, which includes visual acuity testing, refraction, and retinoscopy. Imaging tests such as MRI or CT scans may also be used to evaluate the cause of the swelling.

Treatment of papilledema depends on the underlying cause of the condition. In cases where high blood pressure is the cause, medication to lower blood pressure may be prescribed. In other cases, surgery or other interventions may be necessary to relieve pressure on the brain and reduce swelling in the optic disc.

It's important for individuals with papilledema to work closely with their healthcare provider to monitor and manage their condition, as untreated papilledema can lead to permanent vision loss.

There are several types of ocular albinism, including:

1. Oculocutaneous albinism (OCA) - This is the most common form of ocular albinism and affects both the eyes and skin. It is caused by mutations in the TYR gene, which codes for the enzyme tyrosinase, which is involved in the production of melanin.
2. Hermansky-Pudlak syndrome (HPS) - This is a rare form of ocular albinism that affects both the eyes and platelets. It is caused by mutations in the HPS gene, which codes for the protein hermansky-pudlak syndrome, which is involved in the production of melanin.
3. Juvenile macular degeneration (JMD) - This is a rare form of ocular albinism that affects only the eyes and is caused by mutations in the RPE65 gene, which codes for the protein RPE65, which is involved in the production of melanin.

The symptoms of ocular albinism can vary depending on the type and severity of the condition, but they may include:

* Poor visual acuity (blurred vision)
* Sensitivity to light (photophobia)
* Difficulty seeing colors and fine details
* Eye movements that are slow or uncoordinated
* Increased risk of eye problems such as cataracts, glaucoma, and retinal detachment
* Skin that is pale or freckled

There is no cure for ocular albinism, but treatment options may include glasses or contact lenses to improve vision, medication to reduce the risk of eye problems, and surgery to correct eye alignment or remove cataracts. Early diagnosis and treatment can help manage the symptoms and prevent complications.

Esotropia is often diagnosed in children, and it can affect one or both eyes. Treatment for esotropia usually involves glasses or contact lenses to correct vision problems, as well as exercises to strengthen the muscles that control eye movement. In some cases, surgery may be necessary to realign the eyes.

Esotropia can also be associated with other conditions, such as craniosynostosis (a condition where the bones of the skull fuse together too early), or Down syndrome. It is important for parents and caregivers to be aware of the signs of esotropia, such as crossing or turning of the eyes, and to seek medical attention if they suspect that their child may have this condition. Early diagnosis and treatment can help prevent long-term vision problems and improve the overall quality of life for children with esotropia.

1. Conjunctivitis: This is an infection of the conjunctiva, which is the thin membrane that covers the white part of the eye and the inside of the eyelids. It is often caused by Streptococcus pneumoniae or Haemophilus influenzae bacteria.
2. Corneal ulcers: These are open sores that develop on the surface of the cornea, which is the clear dome-shaped surface at the front of the eye. Corneal ulcers can be caused by a variety of bacteria, including Staphylococcus aureus and Streptococcus pyogenes.
3. Endophthalmitis: This is an infection that occurs inside the eye, often as a complication of cataract surgery or other types of ocular surgery. It can be caused by a variety of bacteria, including Staphylococcus aureus and Streptococcus epidermidis.
4. Keratitis: This is an infection of the cornea that can be caused by a variety of bacteria, including Pseudomonas aeruginosa and Acinetobacter baumannii.
5. Retinitis: This is an infection of the retina, which is the layer of tissue at the back of the eye that senses light and sends visual signals to the brain. Retinitis can be caused by a variety of bacteria, including Haemophilus influenzae and Streptococcus pneumoniae.

Bacterial eye infections can cause a range of symptoms, including redness, swelling, discharge, pain, and blurred vision. Treatment typically involves antibiotic eye drops or ointments, and in more severe cases, oral antibiotics may be prescribed. It is important to seek medical attention if you experience any symptoms of a bacterial eye infection, as early treatment can help prevent complications and improve outcomes.

1. Infection: Bacterial or viral infections can develop after surgery, potentially leading to sepsis or organ failure.
2. Adhesions: Scar tissue can form during the healing process, which can cause bowel obstruction, chronic pain, or other complications.
3. Wound complications: Incisional hernias, wound dehiscence (separation of the wound edges), and wound infections can occur.
4. Respiratory problems: Pneumonia, respiratory failure, and atelectasis (collapsed lung) can develop after surgery, particularly in older adults or those with pre-existing respiratory conditions.
5. Cardiovascular complications: Myocardial infarction (heart attack), cardiac arrhythmias, and cardiac failure can occur after surgery, especially in high-risk patients.
6. Renal (kidney) problems: Acute kidney injury or chronic kidney disease can develop postoperatively, particularly in patients with pre-existing renal impairment.
7. Neurological complications: Stroke, seizures, and neuropraxia (nerve damage) can occur after surgery, especially in patients with pre-existing neurological conditions.
8. Pulmonary embolism: Blood clots can form in the legs or lungs after surgery, potentially causing pulmonary embolism.
9. Anesthesia-related complications: Respiratory and cardiac complications can occur during anesthesia, including respiratory and cardiac arrest.
10. delayed healing: Wound healing may be delayed or impaired after surgery, particularly in patients with pre-existing medical conditions.

It is important for patients to be aware of these potential complications and to discuss any concerns with their surgeon and healthcare team before undergoing surgery.

Definition: Aphakia is a congenital or acquired condition characterized by the absence of the crystalline lens in one or both eyes. It can be classified into different types based on the severity and location of the defect.

Types of Aphakia:

1. Microphthalmia: This type of aphakia is characterized by a small eye that may or may not have a lens.
2. Anophthalmia: This is the most severe form of aphakia where one or both eyes are completely absent.
3. Coloboma: This type of aphakia is characterized by a hole in one of the structures of the eye, such as the iris or retina.

Causes: Aphakia can be caused by genetic mutations, acquired injuries, or infections during pregnancy or childhood. Some of the known causes of aphakia include:

1. Genetic disorders: Certain genetic conditions, such as Turner syndrome, can increase the risk of developing aphakia.
2. Infections: Infections such as rubella or toxoplasmosis during pregnancy can increase the risk of aphakia in the developing fetus.
3. Trauma: Injuries to the eye or head can cause aphakia, especially if they occur during childhood.
4. Tumors: Certain tumors, such as retinoblastoma, can cause aphakia if left untreated.

Symptoms: The symptoms of aphakia can vary depending on the severity of the condition and the age of onset. Some common symptoms include:

1. Blindness or vision loss in one or both eyes
2. Abnormal head positioning or posture
3. Difficulty with depth perception
4. Squinting or tilting the head to see objects clearly
5. Increased sensitivity to light
6. Lazy eye (amblyopia)
7. Poor pupillary reflex
8. Abnormal retinal development
9. Increased risk of other ocular abnormalities, such as cataracts or glaucoma

Diagnosis: Aphakia can be diagnosed through a comprehensive eye exam, including a visual acuity test, refraction test, and ophthalmoscopy. Imaging tests, such as ultrasound or MRI, may also be used to evaluate the structure of the eye and detect any underlying conditions.

Treatment: The treatment for aphakia depends on the severity of the condition and the age of onset. Some possible treatments include:

1. Glasses or contact lenses: To correct refractive errors and improve vision.
2. Patching: To strengthen the weaker eye and improve amblyopia.
3. Atropine therapy: To reduce the amount of accommodation and improve alignment of the eyes.
4. Orthoptic exercises: To improve eye movement and alignment.
5. Surgery: To correct refractive errors, align the eyes properly, or remove any cataracts or other ocular abnormalities.
6. Prosthetic implantation: In some cases, a prosthetic eye may be recommended to restore the natural appearance of the eye and improve vision.

Prognosis: The prognosis for aphakia varies depending on the severity of the condition and the age of onset. In general, early diagnosis and treatment can improve the chances of successful management and a good visual outcome. However, some individuals with aphakia may experience long-term vision loss or other complications, such as amblyopia or glaucoma. Regular follow-up with an eye care professional is important to monitor the condition and adjust treatment as needed.

Synonyms for Aphakia, postcataract include:

* Postoperative aphakia
* Postcataract aphakia
* Aphakic vision loss
* Blindness following cataract surgery

Causes and risk factors for Aphakia, postcataract:

* Cataract surgery: The most common cause of aphakia, postcataract is complications from cataract surgery. During the procedure, the natural lens of the eye may be damaged or removed accidentally.
* Infection: Infections after cataract surgery can cause inflammation and damage to the eye, leading to aphakia.
* Vitreous loss: During cataract surgery, the vitreous gel in the eye may be disturbed or lost, leading to vision loss.

Symptoms of Aphakia, postcataract:

* Blindness or vision loss
* Difficulty seeing objects clearly
* Double vision or ghosting
* Sensitivity to light
* Reduced peripheral vision

Diagnosis and treatment of Aphakia, postcataract:

* Comprehensive eye exam: An ophthalmologist will perform a comprehensive eye exam to determine the cause of the aphakia and assess the extent of vision loss.
* Visual acuity testing: The ophthalmologist will perform visual acuity tests to measure the patient's ability to see objects clearly.
* Retinal imaging: Imaging tests such as ultrasound or MRI may be used to evaluate the retina and diagnose any underlying conditions.
* Glasses or contact lenses: In some cases, glasses or contact lenses may be prescribed to improve vision.
* Intracorneal implant: An intracorneal implant may be recommended to improve vision in cases where the natural lens has been removed and there is no cataract present.
* Corneal transplant: In severe cases of aphakia, a corneal transplant may be necessary to restore vision.

Prevention of Aphakia, postcataract:

* Early detection and treatment of cataracts: Regular eye exams can help detect cataracts early, which can improve the chances of preserving vision and avoiding aphakia.
* Proper follow-up care after cataract surgery: Patients who have undergone cataract surgery should follow their postoperative instructions carefully and attend follow-up appointments to ensure that any complications are detected and treated promptly.
* Preventing eye injuries: Protective eyewear can help prevent eye injuries, which can lead to aphakia.

Prognosis of Aphakia, postcataract:
The prognosis for aphakia after cataract surgery is generally good if the condition is detected and treated promptly. With appropriate treatment, many patients can regain some or all of their vision. However, in severe cases or those with complications, the prognosis may be poorer.

It's important to note that aphakia is a rare complication of cataract surgery, and the vast majority of patients who undergo the procedure do not experience this condition. If you have undergone cataract surgery and are experiencing any unusual symptoms, it is important to seek medical attention promptly to ensure proper diagnosis and treatment.

There are several causes of hemianopsia, including:

1. Stroke or cerebral vasculitis: These conditions can damage the occipital lobe and result in hemianopsia.
2. Brain tumors: Tumors in the occipital lobe can cause hemianopsia by compressing or damaging the visual pathways.
3. Traumatic brain injury: A head injury can cause damage to the occipital lobe and result in hemianopsia.
4. Cerebral palsy: This condition can cause brain damage that leads to hemianopsia.
5. Multiple sclerosis: This autoimmune disease can cause damage to the visual pathways and result in hemianopsia.

Symptoms of hemianopsia may include:

1. Blindness or impaired vision in one side of both eyes.
2. Difficulty recognizing objects or people on one side of the visual field.
3. Inability to see objects that are peripheral to the affected side.
4. Difficulty with depth perception and spatial awareness.
5. Eye movements that are abnormal or restricted.

Diagnosis of hemianopsia typically involves a comprehensive eye exam, including visual acuity testing, visual field testing, and imaging studies such as MRI or CT scans to evaluate the brain. Treatment options for hemianopsia depend on the underlying cause and may include:

1. Glasses or contact lenses to correct refractive errors.
2. Prism lenses to realign the visual image.
3. Visual therapy to improve remaining vision.
4. Medications to treat underlying conditions such as multiple sclerosis or brain tumors.
5. Surgery to repair damaged blood vessels or relieve pressure on the brain.

It is important to note that hemianopsia can significantly impact daily life and may affect an individual's ability to perform certain tasks, such as driving or reading. However, with proper diagnosis and treatment, many people with hemianopsia are able to adapt and lead fulfilling lives.

The exact cause of optic nerve glioma is not known, but it is thought to be related to genetic mutations that occur during fetal development. The tumor typically grows slowly over several years, and may not cause any symptoms in the early stages. As the tumor grows, it can press on the optic nerve and cause vision loss and other symptoms.

There are several types of optic nerve glioma, including:

1. Pilocytic astrocytoma: This is the most common type of optic nerve glioma and typically affects children. It is a slow-growing tumor that usually develops in the optic nerve near the point where it connects to the brain.
2. Fibrillary astrocytoma: This type of optic nerve glioma is less common and tends to grow more quickly than pilocytic astrocytoma. It can occur in both children and adults.
3. Anaplastic astrocytoma: This is the least common and most aggressive type of optic nerve glioma. It typically affects adults and grows rapidly, causing significant vision loss and other symptoms.

The diagnosis of optic nerve glioma is based on a combination of imaging studies such as MRI and CT scans, and tissue biopsy. Treatment options for optic nerve glioma depend on the type and location of the tumor, as well as the patient's age and overall health. Surgery is often the first line of treatment, followed by radiation therapy and chemotherapy as needed. In some cases, clinical trials may also be an option.

Prognosis for optic nerve glioma varies depending on the type and location of the tumor, as well as the patient's age and overall health. In general, the prognosis is better for pilocytic astrocytoma than for fibrillary or anaplastic astrocytoma. However, even with treatment, vision loss can occur in many cases.

In summary, optic nerve glioma is a rare and complex condition that requires careful evaluation and management by a multidisciplinary team of medical professionals. While the prognosis varies depending on the type and location of the tumor, early diagnosis and treatment are critical to improving outcomes for patients with this condition.

It is important to note that this condition can be caused by various factors such as diabetes, high blood pressure, and certain medications. It can also be a symptom of other underlying conditions such as carotid artery disease or aneurysm.

Causes:

* Reduced blood flow to the optic nerve due to various factors such as diabetes, high blood pressure, and certain medications
* Other underlying conditions such as carotid artery disease or aneurysm

Symptoms:

* Vision loss or blindness in one or both eyes
* Blurred vision or double vision
* Loss of peripheral vision
* Sensitivity to light

Diagnosis:

* Dilated eye exam
* Imaging tests such as MRI or CT scans
* Blood tests to check for underlying conditions such as diabetes or high blood pressure

Treatment:

* Treatment of underlying conditions such as diabetes or high blood pressure
* Medications to improve blood flow to the optic nerve
* Surgery to repair any blockages in the carotid artery or other underlying conditions.

Some common types of choroid neoplasms include:

1. Choroidal melanoma: A malignant tumor that arises from the pigment-producing cells of the choroid. It is the most common type of primary intraocular cancer and can spread to other parts of the body if left untreated.
2. Choroidal hemangioma: A benign tumor that arises from the blood vessels of the choroid. It can cause changes in vision and may require treatment to prevent complications.
3. Choroidal naevus: A benign growth that occurs in the choroid and can be inherited. It is usually asymptomatic but can sometimes cause changes in vision.
4. Other rare types of choroid neoplasms include choroidal lymphoma, choroidal osteochondromatosis, and choroidal metastasis (metastasis of cancer from another part of the body to the choroid).

Choroid neoplasms can be diagnosed using a variety of tests, including imaging studies such as ultrasound, CT or MRI scans, and visual field testing. Treatment options vary depending on the type and location of the neoplasm, and may include observation, laser therapy, photodynamic therapy, or surgery.

Overall, choroid neoplasms are complex and varied conditions that require careful evaluation and treatment by an ophthalmologist or other eye care professional to prevent complications and preserve vision.

Photophobia can be caused by various factors, including:

1. Eye conditions like cataracts, glaucoma, or retinal detachment
2. Medications like tranquilizers, antidepressants, or antihistamines
3. Head injuries or brain disorders
4. Chronic diseases such as multiple sclerosis or migraines
5. Vitamin deficiencies like vitamin A or B12
6. Exposure to certain chemicals or toxins

Symptoms of photophobia may include:

1. Discomfort or pain in the eyes when exposed to light
2. Blurred vision or sensitivity to glare
3. Difficulty seeing in bright environments
4. Headaches or migraines triggered by light exposure
5. Nausea or dizziness
6. Sensitivity to light that worsens over time

Diagnosis of photophobia typically involves a comprehensive eye exam to rule out any underlying eye conditions. Medical history and lifestyle factors may also be considered to identify potential causes. Treatment options for photophobia depend on the underlying cause, but may include:

1. Eyewear with tinted lenses or UV protection
2. Medications to reduce light sensitivity or alleviate symptoms
3. Adjustments to lighting environments
4. Lifestyle changes like avoiding bright lights, wearing sunglasses, or using a brimmed hat
5. Treatment of underlying conditions or diseases causing photophobia.

In summary, photophobia is a condition characterized by an excessive sensitivity to light, which can cause various discomforts and symptoms. Identifying the underlying cause through comprehensive diagnosis and implementing appropriate treatment options can help alleviate these symptoms and improve quality of life for individuals experiencing photophobia.

Some common symptoms of corneal edema include:

* Blurred vision
* Haziness or clouding of the cornea
* Increased sensitivity to light
* Redness or discharge in the eye
* Pain or discomfort in the eye

Corneal edema can be diagnosed through a comprehensive eye exam, which may include a visual acuity test, dilated eye exam, and imaging tests such as cornea scans or ultrasound. Treatment for corneal edema depends on the underlying cause and may involve antibiotics, anti-inflammatory medications, or other therapies to reduce swelling and promote healing. In some cases, surgery may be necessary to remove scar tissue or improve drainage of fluid from the eye.

If left untreated, corneal edema can lead to more serious complications such as corneal ulcers or vision loss. Therefore, it is important to seek medical attention if you experience any symptoms of corneal edema to prevent any further damage and ensure proper treatment.

1. Retinitis pigmentosa (RP): a group of degenerative diseases that affect the retina and cause progressive vision loss.
2. Leber congenital amaurosis (LCA): a rare inherited disorder that causes blindness or severe visual impairment at birth or in early childhood.
3. Stargardt disease: a genetic disorder that affects the retina and can cause progressive vision loss, usually starting in childhood.
4. Juvenile macular degeneration (JMD): a group of inherited conditions that affect the macula, the part of the retina responsible for central vision.
5. Persistent hyperplastic primary vitreous (PHPV): a rare inherited condition where abnormal development of the eye can cause vision loss or blindness.
6. Anophthalmia/microphthalmia: a rare inherited condition where one or both eyes are absent or severely underdeveloped.
7. ocular albinism: a genetic condition that affects the development of pigment in the eye, leading to visual impairment and increased risk of eye conditions such as cataracts and glaucoma.
8. Peter's anomaly: a rare inherited condition where there is an abnormal development of the cornea and lens of the eye, leading to vision loss or blindness.
9. cone-rod dystrophy: a group of inherited conditions that affect the retina and can cause progressive vision loss, usually starting in childhood.
10. Retinal dystrophy: a general term for a group of inherited disorders that affect the retina and can cause progressive vision loss, usually starting in adulthood.

These are just a few examples of hereditary eye diseases. There are many other conditions that can be inherited and affect the eyes. Genetic testing and counseling can help identify the risk of inheriting these conditions and provide information on how to manage and treat them.

Open-angle glaucoma can lead to damage to the optic nerve, which can cause vision loss and even blindness if left untreated. It is important for individuals at risk for open-angle glaucoma to receive regular eye exams to monitor their eye pressure and prevent any potential vision loss.

Risk factors for developing open-angle glaucoma include:

* Increasing age
* Family history of glaucoma
* African or Hispanic ancestry
* Previous eye injuries or surgeries
* Long-term use of corticosteroid medications
* Diabetes or other health conditions that can damage blood vessels.

There are several treatment options available for open-angle glaucoma, including:

* Eye drops to reduce eye pressure
* Oral medications to reduce eye pressure
* Laser surgery to improve drainage of fluid from the eye
* Incisional surgery to improve drainage of fluid from the eye.

It is important for individuals with open-angle glaucoma to work closely with their eye care professional to determine the best course of treatment and monitor their condition regularly.

Examples:

* Pupillary anomalies: Abnormalities in the size, shape, or position of the pupil.
* Pupillary block: A condition where the pupil is unable to open properly due to a blockage or obstruction.
* Pupillary dilation: The widening of the pupil, which can be a sign of certain medical conditions.
* Pupillary constriction: The narrowing of the pupil, which can be a sign of other medical conditions.

Symptoms:

* Difficulty seeing or blurred vision
* Sensitivity to light
* Eye pain or discomfort
* Redness or swelling of the eye
* Difficulty moving the eyes

Diagnosis:

* Comprehensive eye exam
* Pupillary reactivity test: Measures how responsive the pupils are to light.
* Ophthalmoscopy: Examines the interior of the eye, including the retina and optic nerve.

Treatment:

* Glasses or contact lenses to correct refractive errors
* Medication to treat underlying conditions such as infection or inflammation
* Surgery to remove blockages or repair damaged tissue
* Pupillary dilators to widen the pupil and improve vision.

Also known as: Corneal inflammation, Eye inflammation, Keratoconjunctivitis, Ocular inflammation.

Some common types of perceptual disorders include:

1. Visual perceptual disorders: These disorders affect an individual's ability to interpret and make sense of visual information from the environment. They can result in difficulties with recognizing objects, perceiving depth and distance, and tracking movement.
2. Auditory perceptual disorders: These disorders affect an individual's ability to interpret and make sense of sound. They can result in difficulties with hearing and understanding speech, as well as distinguishing between different sounds.
3. Tactile perceptual disorders: These disorders affect an individual's ability to interpret and make sense of touch. They can result in difficulties with recognizing objects through touch, as well as interpreting tactile sensations such as pain, temperature, and texture.
4. Olfactory perceptual disorders: These disorders affect an individual's ability to interpret and make sense of smells. They can result in difficulties with identifying different odors and distinguishing between them.
5. Gustatory perceptual disorders: These disorders affect an individual's ability to interpret and make sense of tastes. They can result in difficulties with identifying different flavors and distinguishing between them.
6. Balance and equilibrium disorders: These disorders affect an individual's ability to maintain balance and equilibrium. They can result in difficulties with standing, walking, and maintaining posture.

Perceptual disorders can have a significant impact on an individual's daily life, making it difficult to perform everyday tasks and activities. Treatment for perceptual disorders often involves a combination of sensory therapy, behavioral therapy, and assistive technologies. The goal of treatment is to help the individual compensate for any impairments in sensory processing and improve their ability to function in daily life.

Causes: There are several causes of night blindness, including:

1. Vitamin A deficiency: Vitamin A is essential for the health of the retina, and a deficiency can lead to night blindness.
2. Retinitis pigmentosa: This is a group of inherited conditions that can cause progressive damage to the retina and result in night blindness.
3. Cataracts: A cataract can cause a person to become night blind by blocking the light that enters the eye.
4. Glaucoma: This is a group of eye conditions that can damage the optic nerve and lead to vision loss, including night blindness.
5. Other medical conditions: Certain medical conditions such as diabetes, multiple sclerosis, and stroke can cause night blindness.

Symptoms: The symptoms of night blindness can vary depending on the underlying cause, but common symptoms include:

1. Difficulty seeing in low light environments
2. Blind spots or missing areas of vision
3. Sensitivity to light
4. Glare or halos around lights
5. Difficulty adjusting to changes in light levels

Diagnosis: Night blindness is typically diagnosed through a comprehensive eye exam, which may include a visual acuity test, refraction test, and retinal examination. Imaging tests such as an OCT scan or retinal photography may also be used to evaluate the retina and optic nerve.

Treatment: The treatment of night blindness depends on the underlying cause. For example, vitamin A supplements may be prescribed for a vitamin A deficiency, while cataract surgery may be recommended for cataracts. In some cases, no treatment may be necessary, and the condition may resolve on its own over time.

Prevention: While some cases of night blindness are unavoidable, there are steps you can take to reduce your risk of developing the condition. These include:

1. Maintaining a healthy diet that includes foods rich in vitamin A and other essential nutrients for eye health.
2. Wearing sunglasses with UV protection to protect your eyes from excessive sunlight.
3. Avoiding smoking and excessive alcohol consumption, which can damage the optic nerve and retina.
4. Getting regular eye exams to detect any underlying eye problems early on.
5. Wearing protective eyewear when engaging in activities that could potentially harm your eyes, such as sports or working with hazardous materials.

There are several subtypes of neovascular glaucoma, including:

1. Rubeosis iridis: This is a type of neovascular glaucoma that occurs when new blood vessels grow on the surface of the iris.
2. Uveitic glaucoma: This is a type of neovascular glaucoma that occurs in people with uveitis, an inflammatory condition that affects the inner layers of the eye.
3. Chronic ocular inflammation: This is a type of neovascular glaucoma that occurs when there is chronic inflammation in the eye, leading to the growth of new blood vessels.

Neovascular glaucoma is typically diagnosed with a comprehensive eye exam, which includes a visual acuity test, dilated eye exam, and imaging tests such as ultrasound or optical coherence tomography (OCT). Treatment for neovascular glaucoma usually involves medication to reduce inflammation and pressure in the eye, as well as laser surgery to destroy the new blood vessels. In some cases, a trabeculectomy, or filter surgery, may be performed to drain excess fluid from the eye and reduce pressure.

There are several types of eye hemorrhages, including:

1. Subretinal hemorrhage: This type of hemorrhage occurs between the retina and the choroid, and can cause vision loss if the bleeding is severe.
2. Intravitreal hemorrhage: This type of hemorrhage occurs within the vitreous humor, the gel-like substance inside the eye. It can cause clouding of the lens and vision loss.
3. Retinal hemorrhage: This type of hemorrhage occurs on the surface of the retina and can cause vision loss if the bleeding is severe.
4. Choroidal hemorrhage: This type of hemorrhage occurs within the choroid, the layer of blood vessels between the sclera and retina. It can cause vision loss if the bleeding is severe.

Eye hemorrhages can be diagnosed through a comprehensive eye exam, which includes visual acuity testing, dilated eye examination, and imaging tests such as fluorescein angiography or optical coherence tomography (OCT). Treatment for eye hemorrhages depends on the underlying cause and severity of the condition. In some cases, no treatment may be necessary, while in other cases, medication or surgery may be required to prevent further bleeding and restore vision.

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Ocular hypertension refers to an increase in the pressure within the eye, which can lead to various eye problems if left untreated. It is a common condition that affects millions of people worldwide. In this article, we will provide a comprehensive overview of ocular hypertension, including its definition, causes, symptoms, diagnosis, and treatment options.

What is Ocular Hypertension?
-------------------------

Ocular hypertension is a condition characterized by an increase in the pressure within the eye, which can cause damage to the eye's delicate structures, such as the retina and optic nerve. The normal pressure range for the eye is between 10-21 mmHg, and anything above this range is considered hypertensive.

Causes of Ocular Hypertension
---------------------------

There are several factors that can contribute to the development of ocular hypertension. These include:

* Genetics: People with a family history of glaucoma are more likely to develop ocular hypertension.
* Age: The risk of developing ocular hypertension increases with age, especially after the age of 40.
* Race: African Americans are at a higher risk of developing ocular hypertension than other races.
* Other health conditions: Certain health conditions, such as diabetes and high blood pressure, can increase the risk of developing ocular hypertension.
* Medications: Long-term use of certain medications, such as steroids, can increase eye pressure.

Symptoms of Ocular Hypertension
---------------------------

Ocular hypertension is often asymptomatic, meaning that there are no noticeable symptoms. However, some people may experience the following symptoms:

* Blurred vision
* Eye pain or discomfort
* Redness of the eye
* Seeing halos around lights
* Nausea and vomiting

Diagnosis of Ocular Hypertension
------------------------------

Ocular hypertension can be diagnosed with a comprehensive eye exam. The exam includes:

* Visual acuity test: This test measures how well you can see at different distances.
* Dilated eye exam: This test allows your doctor to examine the inside of your eyes and check for any signs of ocular hypertension.
* Tonometry: This test measures the pressure inside your eyes.
* Ophthalmoscopy: This test allows your doctor to examine the back of your eyes and look for any signs of ocular hypertension.

Treatment of Ocular Hypertension
-----------------------------

There is no cure for ocular hypertension, but there are several treatments that can help manage the condition and prevent vision loss. These include:

* Eye drops: Medicated eye drops can be used to lower eye pressure.
* Oral medications: Oral medications, such as carbonic anhydrase inhibitors, can be used to lower eye pressure.
* Laser surgery: Laser surgery can be used to increase the drainage of fluid from the eye and lower eye pressure.
* Filtering surgery: Filtering surgery can be used to remove the vitreous gel and reduce eye pressure.

Prevention of Ocular Hypertension
-----------------------------

There is no sure way to prevent ocular hypertension, but there are several steps you can take to lower your risk of developing the condition. These include:

* Getting regular eye exams: Regular eye exams can help detect ocular hypertension early, when it is easier to treat.
* Maintaining a healthy weight: Being overweight or obese can increase your risk of developing ocular hypertension.
* Eating a healthy diet: A diet rich in fruits and vegetables can help keep your eyes healthy.
* Exercising regularly: Regular exercise can help improve blood flow and reduce eye pressure.
* Wearing protective eyewear: Wearing protective eyewear, such as sunglasses, can help protect your eyes from UV radiation and reduce your risk of developing ocular hypertension.

Prognosis of Ocular Hypertension
-----------------------------

The prognosis for ocular hypertension is generally good if the condition is detected and treated early. However, if left untreated, ocular hypertension can lead to vision loss and even blindness. It is important to seek medical attention if you experience any symptoms of ocular hypertension, such as blurred vision, eye pain, or seeing flashes of light.

Treatment for ocular hypertension usually involves medication to lower eye pressure. In some cases, laser surgery may be necessary to improve drainage of fluid from the eye. If left untreated, ocular hypertension can lead to more severe complications, such as glaucoma, which can cause permanent vision loss.

Conclusion
----------

Ocular hypertension is a common condition that can increase your risk of developing glaucoma and other eye problems. While there is no cure for ocular hypertension, early detection and treatment can help prevent complications. By understanding the causes, symptoms, diagnosis, and treatment options for ocular hypertension, you can take steps to protect your vision and maintain good eye health.

FAQs
----

1. Can ocular hypertension be cured?
No, there is no cure for ocular hypertension. However, early detection and treatment can help prevent complications.
2. What are the symptoms of ocular hypertension?
Symptoms of ocular hypertension may include blurred vision, eye pain, seeing flashes of light, and blind spots in your peripheral vision.
3. How is ocular hypertension diagnosed?
Ocular hypertension is typically diagnosed with a comprehensive eye exam, including a visual acuity test, dilated eye exam, and tonometry.
4. Can ocular hypertension lead to other eye problems?
Yes, untreated ocular hypertension can increase your risk of developing glaucoma and other eye problems, such as cataracts and optic nerve damage.
5. What are the treatment options for ocular hypertension?
Treatment for ocular hypertension usually involves medication to lower eye pressure, but in some cases, laser surgery may be necessary.
6. Is ocular hypertension inherited?
Yes, ocular hypertension can be inherited, and certain genetic factors can increase your risk of developing the condition.
7. Can ocular hypertension cause blindness?
Yes, if left untreated, ocular hypertension can lead to blindness due to optic nerve damage or glaucoma.
8. How can I reduce my risk of developing ocular hypertension?
You can reduce your risk of developing ocular hypertension by maintaining a healthy lifestyle, including regular exercise, a balanced diet, and not smoking. It is also important to have regular eye exams, especially if you have a family history of the condition.

Characteristics: People with cortical blindness may have difficulty recognizing objects, navigating their environment, and perceiving light and colors. They may also experience visual hallucinations or distortions. The blindness can be partial or total, and the degree of vision loss can vary widely.

Causes: Cortical blindness can be caused by a variety of factors, including:

* Stroke or brain injury that damages the visual cortex
* Infections such as meningitis or encephalitis that affect the visual cortex
* Genetic disorders such as retinitis pigmentosa or Leber's congenital amaurosis
* Traumatic brain injury
* Tumors or cysts in the visual cortex

Symptoms: Symptoms of cortical blindness can include:

* Difficulty recognizing objects or faces
* Poor spatial awareness and navigation
* Blurred or distorted vision
* Sensitivity to light or glare
* Visual hallucinations or distortions
* Partial or total loss of vision

Diagnosis: Diagnosis of cortical blindness typically involves a comprehensive eye exam, neurological evaluation, and imaging tests such as MRI or CT scans to identify any underlying causes.

Treatment: There is no cure for cortical blindness, but various therapies and strategies can help improve functional vision and independence. These may include:

* Vision rehabilitation therapy to improve visual function and adapt to new ways of seeing
* Assistive technology such as telescopes or closed-circuit television systems to enhance remaining vision
* Training in mobility and orientation skills
* Compensatory strategies for daily activities

Prognosis: The prognosis for cortical blindness varies depending on the underlying cause and severity of the condition. In some cases, partial recovery of vision may be possible with time and therapy, while in other instances, visual function may remain impaired or stable.

Prevention: Prevention of cortical blindness is not always possible, but early detection and treatment of underlying conditions can help reduce the risk of vision loss. Regular eye exams and monitoring by a neurologist or ophthalmologist can also help identify any changes in vision and potential causes of cortical blindness.

Overall, cortical blindness is a complex and rare condition that requires specialized care and rehabilitation to improve functional vision and independence. With the right therapies and strategies, individuals with cortical blindness can lead fulfilling lives and adapt to their new visual reality.

There are many different types of retinal degeneration, each with its own set of symptoms and causes. Some common forms of retinal degeneration include:

1. Age-related macular degeneration (AMD): This is the most common form of retinal degeneration and affects the macula, the part of the retina responsible for central vision. AMD can cause blind spots or distorted vision.
2. Retinitis pigmentosa (RP): This is a group of inherited conditions that affect the retina and can lead to night blindness, loss of peripheral vision, and eventually complete vision loss.
3. Leber congenital amaurosis (LCA): This is a rare inherited condition that causes severe vision loss or blindness at birth or within the first few years of life.
4. Stargardt disease: This is a rare inherited condition that causes progressive vision loss and can lead to blindness.
5. Retinal detachment: This occurs when the retina becomes separated from the underlying tissue, causing vision loss.
6. Diabetic retinopathy (DR): This is a complication of diabetes that can cause damage to the blood vessels in the retina and lead to vision loss.
7. Retinal vein occlusion (RVO): This occurs when a blockage forms in the small veins that carry blood away from the retina, causing vision loss.

There are several risk factors for retinal degeneration, including:

1. Age: Many forms of retinal degeneration are age-related and become more common as people get older.
2. Family history: Inherited conditions such as RP and LCA can increase the risk of retinal degeneration.
3. Genetics: Some forms of retinal degeneration are caused by genetic mutations.
4. Diabetes: Diabetes is a major risk factor for diabetic retinopathy, which can cause vision loss.
5. Hypertension: High blood pressure can increase the risk of retinal vein occlusion and other forms of retinal degeneration.
6. Smoking: Smoking has been linked to an increased risk of several forms of retinal degeneration.
7. UV exposure: Prolonged exposure to UV radiation from sunlight can increase the risk of retinal degeneration.

There are several treatment options for retinal degeneration, including:

1. Vitamin and mineral supplements: Vitamins A, C, and E, as well as zinc and selenium, have been shown to slow the progression of certain forms of retinal degeneration.
2. Anti-vascular endothelial growth factor (VEGF) injections: These medications can help reduce swelling and slow the progression of diabetic retinopathy and other forms of retinal degeneration.
3. Photodynamic therapy: This involves the use of a light-sensitive medication and low-intensity laser light to damage and shrink abnormal blood vessels in the retina.
4. Retinal implants: These devices can be used to restore some vision in people with advanced forms of retinal degeneration.
5. Stem cell therapy: Research is ongoing into the use of stem cells to repair damaged retinal cells and restore vision.

It's important to note that early detection and treatment of retinal degeneration can help to slow or stop the progression of the disease, preserving vision for as long as possible. Regular eye exams are crucial for detecting retinal degeneration in its early stages, when treatment is most effective.

The exact cause of ROP is not known, but it is thought to be related to the immaturity of the retina and the high levels of oxygen in incubators used to care for premature babies. The risk of developing ROP increases with the degree of prematurity, with infants born before 28 weeks gestation being at highest risk.

ROP typically develops in two stages:

1. Stage 1: Early ROP - This stage is characterized by the formation of small blood vessels and immature retinal tissue.
2. Stage 2: Advanced ROP - This stage is characterized by the proliferation of abnormal blood vessels, bleeding, and scarring in the retina.

There are several subtypes of ROP, including:

1. Type 1 ROP: Mildest form of the disease, with few or no complications.
2. Type 2 ROP: More severe form of the disease, with abnormal blood vessel growth and scarring in the retina.
3. Type 3 ROP: Most severe form of the disease, with widespread scarring and bleeding in the retina.

Treatment for ROP typically involves monitoring the infant's eye development closely and applying laser therapy to the affected areas if necessary. In severe cases, surgery may be required to remove abnormal blood vessels or scar tissue.

Prevention of ROP is primarily focused on reducing the risk factors, such as prematurity and oxygen exposure. This includes:

1. Proper management of gestational diabetes to prevent preterm birth.
2. Close monitoring of fetal development and early delivery if necessary.
3. Careful regulation of oxygen levels in incubators to avoid over-oxygenation.
4. Early detection and treatment of infections that can lead to preterm birth.
5. Avoiding excessive use of ophthalmic drugs that can be harmful to the developing retina.

Early detection and timely intervention are crucial for effective management and prevention of ROP. Regular eye exams and screening are necessary to identify the disease in its early stages, when treatment is most effective.

It can occur in various conditions such as:

1. Graves' disease (an autoimmune disorder causing hyperthyroidism)
2. Toxic amblyopia (poisoning caused by organophosphate pesticides or other toxins)
3. Orbital tumors (such as aneurysmal bone cysts or lymphoma)
4. Inflammatory conditions such as endogenous fungal infections or tuberculosis
5. Trauma (head injury, blunt force trauma to the eye socket)
6. Neuromuscular disorders (such as myasthenia gravis)
7. Ischemic optic neuropathy (reduced blood flow to the nerve that carries visual information from the eye to the brain)
8. Anophthalmia/microphthalmia (absence or underdevelopment of one or both eyes)

Symptoms of exophthalmos may include:

* Bulging eyes
* Diplopia (double vision)
* Ptosis (drooping eyelid)
* Eye pain or discomfort
* Redness and swelling of the conjunctiva (the mucous membrane covering the white part of the eye)
* Vision changes such as blurred vision or loss of peripheral vision

Treatment options for exophthalmos depend on the underlying cause, but may include medication, surgery, or a combination of both.

Symptoms: blurred vision, halos around lights, redness and pain in the eye, nausea and vomiting, and sensitivity to light.

Diagnosis: a comprehensive eye exam, including measurements of intraocular pressure (IOP) and assessment of the angle of the eye.

Treatment: may include medication to reduce IOP, laser or surgical treatment to improve drainage, and in some cases, vitrectomy (removal of the vitreous gel).

Prognosis: with prompt and appropriate treatment, vision can be preserved. However, if left untreated, angle-closure glaucoma can lead to permanent vision loss.

Etiology: can be caused by a variety of factors, including age-related changes, cataract surgery, trauma, and inflammation.

Prevalence: is more common in certain populations, such as those of Asian descent, and in those with a family history of the condition.

The exact cause of retinal vasculitis is not known, but it is believed to be an autoimmune disorder, meaning that the body's immune system mistakenly attacks its own tissues. It can occur at any age but is more common in adults between 30 and 60 years old.

Symptoms of retinal vasculitis include:

1. Blurred vision or blind spots
2. Floaters (specks or cobwebs in vision)
3. Flashes of light
4. Redness and pain in the eye
5. Sensitivity to light
6. Blindness in one or both eyes

Retinal vasculitis is diagnosed through a comprehensive eye exam, including a visual acuity test, dilated eye exam, and imaging tests such as fluorescein angiography and optical coherence tomography (OCT).

Treatment options for retinal vasculitis include:

1. Corticosteroids to reduce inflammation
2. Immunosuppressive drugs to suppress the immune system
3. Anti-vascular endothelial growth factor (VEGF) injections to prevent the growth of new blood vessels
4. Photodynamic therapy to damage and shrink the abnormal blood vessels
5. Vitrectomy, a surgical removal of the vitreous gel and any blood or scar tissue in the eye

The prognosis for retinal vasculitis varies depending on the severity and location of the disease, but it can lead to significant vision loss if left untreated. It is important to seek medical attention if symptoms persist or worsen over time.

Symptoms of choroiditis may include blurred vision, sensitivity to light, redness and pain in the affected eye, and seeing floaters or flashes of light. In severe cases, it can lead to retinal detachment, which can cause permanent vision loss if not treated promptly.

Diagnosis of choroiditis typically involves a comprehensive eye exam, including a visual acuity test, dilated eye exam, and imaging tests such as fluorescein angiography or optical coherence tomography to evaluate the retina and choroid. Treatment options for choroiditis depend on the underlying cause, but may include antibiotics or anti-inflammatory medications, corticosteroids, and in some cases, surgery.

Angioid streaks can be detected during an eye exam using a specialized microscope called a fundus camera. If the streaks are caused by diabetic retinopathy or other underlying conditions, treatment may involve managing the underlying condition to prevent further damage to the blood vessels in the retina. In some cases, laser surgery may be recommended to seal off leaking blood vessels and prevent further bleeding.

In summary, Angioid streaks are a sign of damage to the blood vessels in the retina and can be a warning sign of more serious underlying conditions such as diabetic retinopathy or hypertensive retinopathy. It is important to seek medical attention if you notice any changes in your vision or see flashes of light, as these can be signs of a more serious condition.

In the medical field, telangiectasis may be diagnosed through a physical examination and/or imaging tests such as ultrasound or MRI. Treatment options for telangiectasis depend on the underlying cause of the condition but may include topical creams or ointments, laser therapy, or lifestyle changes.

Some synonyms for telangiectasis are: spider veins, telangiectatic vessels, and spider naevi.

Note: Telangiectasis is not to be confused with telengectasis which is a condition where the blood vessels in the lung become dilated and can lead to pulmonary embolism.

There are many different types of uveal diseases, including:

1. Uveitis: This is inflammation of the uvea, which can be caused by a variety of factors such as infection, injury, or autoimmune disorders.
2. Iridocyclitis: This is inflammation of the iris and ciliary body.
3. Choroiditis: This is inflammation of the choroid layer of the uvea.
4. Retinal vein occlusion: This is a blockage of the veins that carry blood away from the retina, which can cause vision loss.
5. Macular edema: This is swelling of the macula, the part of the retina responsible for central vision.
6. Age-related macular degeneration (AMD): This is a condition that affects the macula and can cause vision loss over time.
7. Diabetic retinopathy: This is a complication of diabetes that can cause damage to the blood vessels in the retina and lead to vision loss.
8. Retinal detachment: This is a condition where the retina becomes separated from the underlying tissue, leading to vision loss.
9. Retinal vein thrombosis: This is a blockage of the veins that carry blood away from the retina, which can cause vision loss.
10. Uveal melanoma: This is a type of cancer that affects the uvea and can be potentially life-threatening.

These are just a few examples of uveal diseases, and there are many other conditions that can affect the uvea as well. Treatment options for uveal diseases vary depending on the specific condition and its cause, but may include medications, laser surgery, or other procedures to treat inflammation, reduce swelling, or remove tumors.

... visual acuity. Thus, "normal" visual acuity by no means implies normal vision. The reason visual acuity is very widely used is ... Visual acuity alone thus cannot determine the overall quality of visual function. Visual acuity is a measure of the spatial ... Bach M (2016). "The Freiburg Visual Acuity Test". Visual Functions Committee (25 May 1984). "Visual acuity measurement standard ... So, distant visual acuity of 6/10 and 6/8 with pinhole in the right eye will be: DscOD 6/10 PH 6/8. Distant visual acuity of ...
"Visual acuity and hyperacuity". Investigative Ophthalmology and Visual Science. 14: 570-572. Robert H. Duckman (2006). Visual ... Vernier acuity is a type of visual acuity - more precisely of hyperacuity - that measures the ability to discern a disalignment ... However, vernier acuity decreases more quickly than grating acuity in peripheral vision. Vernier acuity was first explained by ... Because vernier acuity exceeds acuity by far, the phenomenon has been termed hyperacuity. Vernier acuity develops rapidly ...
"Visual Acuity". American Optometric Association. Retrieved 3 December 2015. Romero-Corral, Abel; Caples, Sean; Lopez-Jimenez, ... Individuals with MORM syndrome experience decreased visual acuity, meaning their ability to see distinct sharp lines decreases ... Any further reduction in vision acuity is not observed until the individual is between the ages thirty to forty. Delayed ...
Westheimer, G (1 January 1965). "Visual Acuity". Annual Review of Psychology. 16 (1): 359-380. doi:10.1146/annurev.ps.16.020165 ... Gittings, Neil S.; Fozard, James L. (1986). "Age related changes in visual acuity". Experimental Gerontology. 21 (4-5): 423-433 ... atmospheric aerosols such as dust or air pollution visual acuity of the observer wind (can cause stars to twinkle, and render ... Assuming normal visible acuity and clear skies, it is possible to approximately convert Globe at Night naked eye limiting ...
Visual acuity. A practical measure of binocularity is the binocular summation ratio BSR, which is the ratio of binocular ... By combining the information received in each eye, binocular summation can improve visual acuity, contrast sensitivity, flicker ... Binocular summation refers to the improved visual performance of binocular vision compared to that of monocular vision. The ... Some of the ways in which binocular summation improves binocular visual performance are Brightness perception. The binocularly ...
Their visual acuity may rival some eagles from the genus Aquila and some of the larger falcons as the greatest of all diurnal ... Fox, R., Lehmkuhle, S.W., & Westendorf, D.H. (1976). Falcon visual acuity. Science, 192(4236), 263-265. Chapin, J.P., & Lang, H ... Reymond, L. (1985). Spatial visual acuity of the eagle Aquila audax: a behavioural, optical and anatomical investigation. ... Martial eagles have been noted as remarkable for their extremely keen eyesight (3.0-3.6 times human acuity), partly due to ...
Fox, R; Lehmkuhle, S.; Westendorf, D. (1976). "Falcon visual acuity". Science. 192 (4236): 263-65. Bibcode:1976Sci...192..263F ... the visual acuity of one species has been measured at 2.6 times that of a normal human. Peregrine falcons have been recorded ...
Low vision is defined as a best-corrected visual acuity worse than 0.5 LogMAR but equal or better than 1.3 LogMAR in the better ... When using a LogMAR chart, visual acuity is scored with reference to the logarithm of the minimum angle of resolution, as the ... Blindness is defined as a best-corrected visual acuity worse than 1.3 LogMAR. Bailey IL, Lovie JE. I (1976.) New design ... Bailey IL, Lovie JE (2013). Visual acuity testing. From the laboratory to the clinic. Vision Research 90: 2-9. doi:10.1016/j. ...
Her most notable work was in the area of visual acuity testing where she developed and improved equipment. Sloan received her ... Sloan's lifetime of work in the area of visual acuity testing continues to have many longstanding applications to the work done ... Sloan, Louise L. (1959-12-01). "New test Charts for the Measurement of Visual Acuity at far and Near Distances*". American ... Bailey, Ian L.; Lovie-Kitchin, Jan E. (2013-09-20). "Visual acuity testing. From the laboratory to the clinic". Vision Research ...
... decreased visual acuity; seeing specks or spots; teary eyes; or eye discharge or crusting. On February 25, 2005, Alcon filed a ...
Retrieved 14 July 2010 from Encyclopedia.com "How Far Can the Human Eye See? , Human Visual Acuity , LiveScience". Archived ... Uttal, William R. (2014). A Taxonomy of Visual Processes. Psychology Press. p. 389. ISBN 978-1-317-66895-4. Reike, Fred (2000 ... Cornsweet, Tom (1970). "Chapters 2 and 4". Visual Perception. Harcourt Publishing. Hecht, Selig; Shlaer, Simon; Pirenne, ... to optimise their visual sensitivity. Location - the stimulus was presented to an area of the right eye where there is a high ...
Bailey IL, Lovie JE (2013). "Visual acuity testing. From the laboratory to the clinic". Vision Research. 90: 2-9. doi:10.1016/j ... 2014-04-23). "Effect of acetazolamide on visual function in patients with idiopathic intracranial hypertension and mild visual ... a set of three logMAR charts now widely used for the measurement of visual acuity in vision research and clinical practice. A ... The study uses visual function status to compare two intervention sequences in managing the disease. After seven years of ...
Land MF (1997). "Visual acuity in insects". Annual Review of Entomology. 42: 147-77. doi:10.1146/annurev.ento.42.1.147. PMID ... This visual arrangement is known as neural superposition.: 163-4 Since an image from the compound eye is created from the ... and each rhabdomere is united with those from the six adjacent ommatidia that share the same visual axis. Thus, at the level of ... 161-2 the advantage of this arrangement is that the same visual axis is sampled from a larger area of the eye, increasing ...
Land, Michael F. (January 1997). "Visual Acuity in Insects". Annual Review of Entomology. 42 (1): 147-177. doi:10.1146/annurev. ... "The visual orientation strategies of Mantis religiosa and Empusa fasciata reflect differences in the structure of their visual ... The overlap of the visual field of the two eyes is 40° in nymphs and up to 70° in adults. The pseudopupil, a small, dark region ... The development of the visual system was reviewed by Karl Kral in 2014: while high-contrast cues could be perceived by adults ...
Westheimer, G. (1975). Visual acuity and hyperacuity. Investigative Ophthalmology, 14, 570-572. Gilbert, C., Ito, M., Kapadia, ... M., & Westheimer, G. (2000 ). Interactions between attention, context and learning in primary visual cortex. Vision Research, ...
Hering did seminal work on what we now call hyperacuity: spatial resolution in certain visual tasks that exceeds visual acuity ... following G. Westheimer Westheimer, Gerald (1975). "Visual acuity and hyperacuity". Investigative Ophthalmology and Visual ... In his famous 1899 treatise "On the Limits of Visual Acuity" he summarized empirical data published 1863 by Alfred Wilhelm ... Strasburger, Hans; Huber, Jörg; Rose, David (2018). "Ewald Hering (1899) On the Limits of Visual Acuity: A Translation and ...
Westheimer, G. (1975-08-01). "Editorial: Visual acuity and hyperacuity". Investigative Ophthalmology & Visual Science. 14 (8): ... Visual acuity is measured by the smallest letters that can be distinguished on a chart and is governed by the anatomical ... This defines the resolution limit and the basis of visual acuity. A quite different mechanism operates in hyperacuity, whose ... Strasburger, Hans; Huber, Jörg; Rose, David (2018). "Ewald Hering (1899) On the Limits of Visual Acuity: A Translation and ...
Visual acuity is slightly reduced.[citation needed] Granular corneal dystrophy is caused by a mutation in the TGFBI gene, ...
Primary visual factors: Quantitative: Decreased visual acuity (myopia, hyperopia, astigmatism) Qualitative: Irregular ... After the glass was removed by this method and the cornea healed, Fyodorov found that the boy's visual acuity had improved ... Symons SP, Slomovic AR (August 1994). "Visual acuity, refractive and keratometric results of 140 consecutive radial keratotomy ... Visual acuity is generally improved. The healing corneal wounds consist of newly abutting corneal stroma, fibroblastic cells, ...
Visual hyperacuity is at least 10 times more sensitive than visual acuity, the ability to separate between distinct objects. ... PHP takes advantage of visual hyperacuity, also known as vernier acuity-the ability to identify the misalignment of visual ... Westheimer G. (August 1975). ""Editorial: Visual acuity and hyperacuity"". Invest Ophthalmol. 14 (8): 570-572. PMID 1150397. ( ... Another problem is that minute visual aberrations can be normal and therefore should be distinguished from genuine visual ...
2 Visual Acuity, Contrast Sensitivity". University of Utah. Retrieved 19 July 2009. (CS1 errors: external links, Articles with ... In the study of visual perception, sinusoidal gratings are frequently used to probe the capabilities of the visual system. In ... The spatial-frequency theory refers to the theory that the visual cortex operates on a code of spatial frequency, not on the ... Most neurons in the primary visual cortex respond best when a sine-wave grating of a particular frequency is presented at a ...
A horse has higher acuity through most of the visual field of its eyes than a human has, but does not match the high acuity of ... is then the measurement of visual acuity of the eye. For a human eye with excellent acuity, the maximum theoretical resolution ... Visual acuity, or resolving power, is "the ability to distinguish fine detail" and is the property of cone cells. It is often ... For example, the eye of a bird of prey has much greater visual acuity than a human eye, and in some cases can detect ...
... has superhuman visual acuity. He possesses an above normal intellect, with a capacity for unlimited self-motivated ... 2 suggest that X-51's memories of his time with the Celestials may be skewed, as he experienced visual hallucinations of a ... A Visual History. DK Publishing. pp. 185, 219. ISBN 978-1465455505. Markstein, Don. "Machine Man". Don Markstein's Toonopedia. ...
He also possessed superhuman visual acuity. Skurge was extremely long-lived, aging at a much slower pace than human beings, ... Brevoort, Tom; DeFalco, Tom; Manning, Matthew K.; Sanderson, Peter; Wiacek, Win (2017). Marvel Year By Year: A Visual History. ...
However, some visual abnormalities can remain even where visual acuity is measured at 20/20. This includes localized reductions ... Reduced visual acuity may persist after the fluid has disappeared. The disease is considered of unknown cause. It mostly ... Recovery of visual acuity usually follows. Treatment should be considered if resorption does not occur within 3-4 months, ... It would appear that the presence of the bacteria is well correlated with visual acuity and other retinal findings following an ...
Does weight lifting improve visual acuity? A replication of Gonzalo-Fonrodona and Porras (2013)?» BMC Research Notes 10:362 ... For example, the visual field, which shows concentric reduction, increased up to 5 times in the most acute case, and the image ... It consists of two volumes, the first one published in 1945 focused on visual functions and the second one published in 1950 ... Gonzalo-Fonrodona, I.; Porras, M.A. (2007). «Physiological Laws of Sensory Visual System in Relation to Scaling Power Laws in ...
doi:10.1016/s0079-6611(99)00017-8. Gagnon, Yakir L.; Sutton, Tracey T.; Johnsen, Sönke (November 2013). "Visual acuity in ...
Franciscus Donders introduces the term visual acuity. Guillaume Duchenne describes Duchenne muscular dystrophy. Prosper Ménière ...
Visual acuity is more or less normal. Lattice dystrophy starts as fine branching linear opacities in Bowman's layer in the ... Visual acuity eventually becomes reduced during the second and third decades of life following a progressive superficial haze ... generalized corneal edema and decreased visual acuity. In advanced cases, abnormalities are found in all the layers of the ... Some cause severe visual impairment, while a few cause no vision problems and are diagnosed during a specialized eye ...
... visual acuity. "Sebastien Le Meaux - IPC Profile". International Paralympic Committee. 14 December 2021.{{cite web}}: CS1 maint ...
... high-end systems now offer a resolution that approaches the limit of human visual acuity. LCD projectors have fundamental ... Unable to pay for this himself, Stern became the head of the planetarium division of Viewlex, a mid-size audio-visual firm on ... For especially visual or spatially aware people, this experience can be more educationally beneficial than other demonstrations ...
... visual acuity of 2/60, and/or visual field of less than 5 degrees. F13: visual acuity ranges from 2/60 to 6/60, and/or visual ... The sighted guides who run together with athletes with a visual impairment at the Championship, did not receive a medal. This ...
The horse has a wide field of monocular vision, as well as good visual acuity. Horses have two-color, or dichromatic vision, ... Because the horse's vision is closely tied to behavior, the horse's visual abilities are often taken into account when handling ...
1989 Mar;60(3):188-92 "Visual acuity and optical parameters in progressive-power lenses" Villegas EA, Artal P Optom Vis Sci. ... 2004 May;81(5):350-61 "Will visual discomfort among visual display unit (VDU)" Horgen G, Aarås A, Thoresen M. Optom Vis Sci. ... FAAO Investigative Ophthalmology and Visual Science. 2004;45:2122-2128.) Stepping Up to a New Level: Effects of Blurring Vision ...
Mast, F.W., & Oman, C.M. (2004). Top-Down Processing and Visual Reorientation Illusions in a Virtual Reality Environment. Swiss ... Gough, Harrison & Meschieri, Luigi (1971). Cross-Cultural Study of Age-Related Differences in Perceptual Acuity. Journal of ... Children who showed greater personal independence, verbal articulation, and visual scanning ability were more effective and ... Robinson, J.O. (1972). The Psychology of Visual Illusion. Oxford, England: Hutchinson University Library. Rasmjou, S., Hausmann ...
A stereoscopic rangefinder uses two eyepieces and relies on the operator's visual cortex to merge the two images into a single ... The report concludes with the belief that stereoscopic and coincidence acuities are about equal. Under favourable conditions ...
... visual acuity of 20/60 - limited sight) B3 - partially sighted (visual acuity above 20/60 to 6/60 -more sight than B2) Standing ...
... "create a real acuity in the viewer" : the object of the film is "the construction of the gaze". In Yves, Olivier Zabat "is not ... bringing a singular visual reality to the symbolics of the text". In Arguments, "the challenge" (of voice hearers) "overlaps so ... ", "it is a question of apprehending the film as visual, sound and textual material, with bursts and fades.(...). From one ...
... increased visual acuity, surfaces that seem to ripple, shimmer, or breathe; complex open and closed eye visuals of form ... Sensory effects include visual and auditory hallucinations followed by emotional changes and altered perception of time and ... Noticeable changes to the auditory, visual, and tactile senses may become apparent around 30 minutes to an hour after ingestion ...
... visual acuity and cooperation For accurate (within two prism dioptres) numerical recording that will provide a point of ... ensuring it is in line with their visual axis. By having the patient read out loud the letters at the top of the fixation stick ...
If approached too closely, they generally run in short bursts and stop abruptly to confuse the predator's visual acuity. If ...
Dim lighting minimized visual cues of the two SUs. The central nose-leaf and a semicircular ring of pads construct the nasal ... there is a transient increase in impulse activity which quickly decays due to adaptation and thus strengthens temporal acuity. ... In this study, olfactory and visual stimuli were minimized to ascertain that only thermal cues affected behavioral learning. ...
... with a mix of permanent symptoms such as vastly increased visual acuity along with complete deafness. Those who adapt fully to ...
Herdman, SJ; Schubert MC; Tusa RJ (2001). "Role of Central Preprogramming in Dynamic Visual Acuity With Vestibular Loss". Arch ... during dynamic visual acuity testing. Efference copy within an internal model allows us to grip objects in parallel to a given ... If this is done, one notices that the visual world seems to have "moved" as a result of this passive movement of the eyeball. ... Gyr John W (1972). "Is a Theory of Direct Visual Perception Adequate?". Psychological Bulletin. 77 (4): 246-261 [250]. doi: ...
Swift recommended that riders relax their visual acuity and direct more attention to the tactile interaction between horse and ...
... decreased metamorphopsia is associated with an increase in visual acuity. Quantitative evaluation of metamorphopsia is an ... The cortical mechanism, which was discovered after the retinal mechanism, is affected by perceptual "filling-in" and visual ... Midena, Edoardo; Vujosevic, Stela (2016). "Metamorphopsia: An Overlooked Visual Symptom". Ophthalmic Research. 55 (1): 26-36. ... which assesses a person's ability to any misalignments of visual objects, may permit a more sensitive diagnosis of ...
They found no evidence of improved visual acuity with potential harm. "Peginterferon Alfa-2a (Professional Patient Advice) - ...
A reduction in visual acuity can occur followed neuroretinal breakdown. Associated factors include inflammation, drugs, trauma ... Johnson CA (2003). "The use of a visual illusion to detect glaucoma.". In Andre J, Owens DA, Harvey Jr LO (eds.). Visual ... A formal visual field test is performed. The retinal nerve fiber layer can be assessed with imaging techniques such as optical ... In glaucoma visual field defects result from damage to the retinal nerve fiber layer. Field defects are seen mainly in primary ...
The Men's high jump, F13 was held on January 28 Classification F13 - visual impairment F13: visual acuity ranges from 2/60 to 6 ... may recognise the shape of a hand and have a visual acuity of 2/60 and/or visual field of less than 5 degrees. Key: CR = ... The event also included athletes with a F12 classification - visual impairment F12: ... and/or visual field over 5 degrees and less than 20 degrees. ...
The SB13 category is for swimmers who have minor visual impairment and have high visual acuity. They are required to wear ... The SB12 category is for swimmers who have moderate visual impairment and have a visual field of less than 5 degrees radius. ... The final in this classification took place on 26 August 2021: The SB11 category is for swimmers who have severe visual ...
While agnosics suffer from severe deficits, patients' visual acuity and other visual abilities such as perceiving parts and ... Agnosia Associative visual agnosia Aphasia Visual agnosia Visual space Patient DF David Andrewes (13 May 2013). Neuropsychology ... Visual disturbances and blindness, Visual system, Visual perception). ... Visual apperceptive agnosia is a visual impairment that results in a patient's inability to name objects. ...
The best visual acuity of the human eye at its optical centre (the fovea) is less than 1 arc minute per line pair, reducing ... Other examples are when a human is using eyes to carry out a critical task such as flying (piloting by visual reference), ...
The leading answer is that the high visual acuity that comes with diurnal characteristics isn't needed anymore due to the ... they have been found to have a larger cornea relative to their eye size than diurnal creatures to increase their visual ...
Dawes P, Bishop DV (August 2008). "Maturation of visual and auditory temporal processing in school-aged children". Journal of ... 150 Hz and worsens outside that range The cutoff frequency of the TMTF gives an estimate of temporal acuity (temporal ... has been demonstrated both in the auditory and visual cortices. It has also been shown to be amply manifested in measurements ...
Even though antibiotics can have negative impacts on the retina in high concentrations, the facts that visual acuity worsens in ... In patients with acute endophthalmitis, combined steroid treatment with antibiotics have been found to improve visual outcomes ... and visual outcome". American Journal of Ophthalmology. 160 (1): 17-25.e1. doi:10.1016/j.ajo.2015.04.013. PMID 25892127. Emami ...
The most common symptom is decreased visual acuity in one eye. The diagnosis is made by examination of the eye, using ... A unilateral decrease in visual acuity is the most common symptom of toxoplasmic retinitis. Under ophthalmic examination, ...
Is your visual acuity on a 20/20 scale below the minimum level required by your state? The number of older drivers on the road ... Physical strength, mental acuity, and motor function begin to deteriorate as a person ages, but the degree of decline varies ... Owsley, Cynthia (2001-06-01). "Visual Risk Factors for Crash Involvement in Older Drivers With Cataract". Archives of ... "Effects of aging on optical quality and visual function". Clinical and Experimental Optometry. 99 (6): 518-525. doi:10.1111/cxo ...
... a visual acuity of 2/60 and/or visual field of less than 5 degrees. F13: visual acuity ranges from 2/60 to 6/60, and/or visual ... Key: SB = Season Best The Men's javelin throw, F13 was held on January 29 Classification F13 - visual impairment F12: may ... visual impairment may range from no light perception in either eye, to light perception with inability to recognise the shape ...
They found no lasting decrease in visual acuity. The European Parliament Scientific and Technological Options Assessment (STOA ... 41 (8): 2138 - Investigative Ophthalmology & Visual Science". Investigative Ophthalmology & Visual Science. Iovs.org. 41 (8): ... The Journal of Investigative Ophthalmology and Visual Science published a study that concluded that single exposure of the eye ...
The visual acuity test is used to determine the smallest letters you can read on a standardized chart (Snellen chart) or a card ... Visual acuity is expressed as a fraction.. *The top number refers to the distance you stand from the chart. This is often 20 ... The visual acuity test is used to determine the smallest letters you can read on a standardized chart (Snellen chart) or a card ... For uncorrected visual acuity, you will be asked to remove your glasses or contact lenses and stand or sit 20 feet (6 meters) ...
Unaided distance acuities were recorded, using logarithmically scaled letter charts, on two occasions separated by an interval ... We examined the test-retest reliability of visual acuity in 18 children referred to the Hospital Eye Service with reduced ... Reliability of visual acuity in children with reduced vision Ophthalmic Physiol Opt. 1996 Sep;16(5):447-9. ... The reliability of visual acuity recorded in the better eye closely approximates to that previously found in normal adults, ...
How is visual acuity for both eyes determined? If you have each eye tested, can the scores be combined? ... How is visual acuity (sharpness, power) for both eyes determined? For example, if your right eye is 20/50 and your left eye is ... Each eye has a specific visual acuity. There is no formula to add or combine the two visions and conclude a vision for both ...
This study aimed to analyze the correlation between hybrid lenses and pinhole visual acuity (VA). ,i,Methods,/i,. Patients ... corneal diseases such as keratoconus to determine whether optical aberrations associated with the disease cause their visual ... the best approach to determine whether the correction of residual defocus and irregular astigmatism might improve the visual ... acuity in patients with corneal disorders including keratoconus. ... with Hybrid Contact Lens Visual Acuity in Patients with Visual ...
Conclusion: Unexpectedly, reduction in economic inequality in presenting visual acuity was due to presenting visual acuity ... to specifically modify visual acuity in each group and, consequently, reduce the economic inequality in visual acuity. ... contributor to reduction in economic inequality of presenting visual acuity was deterioration of presenting visual acuity among ... A paradoxical change in economic inequality in presenting visual acuity between 2009 and 2014: a nonuseful decline ...
Palavras-chave : Static visual acuity; dynamic visual acuity; visual abilities; movement perception; visual psychophysics. ... We present a review on the visual ability to discriminate fine details of moving objects (DVA: Dynamic Visual Acuity), showing ... QUEVEDO, Lluïsa; AZNAR-CASANOVA, J. Antonio e SILVA, José Aparecido da. Dynamic visual acuity. Temas psicol. [online]. 2018, ... the most relevant differences, which have been attributed to this visual capacity in comparison to SVA (static visual acuity). ...
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... you can keep track of the progression of your visual acuity. Visual acuity scores of multiple testees could be recorded. Visual ... Visual Acuity Charts. Testing for myopia takes several procedures and the first step is often to check distant visual acuity ... through a visual acuity chart. And the decrement of visual acuity may indicate the development of myopia. ... It is more intuitive since higher values indicate better visual acuity.. In the scores review, score 100 in blue indicates the ...
Best-corrected visual acuity (BCVA) was tested with Landolt rings using an automated device (VA-CAL test). The visual acuity ... The VA-CAL Test Quantifies Improvement of Visual Acuity in Achromatopsia by Means of Short-Wave Cutoff Filter Glasses in Daily ... Translational Relevance The VA-CAL test discovers losses of spatial resolution in the visual acuity space not seen in ... The VA-CAL Test Quantifies Improvement of Visual Acuity in Achromatopsia by Means of Short ...
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It is reduced in size relative to a visual acuity chart and is not intended to be used for visual acuity testing. On the front ... Description Of Item: Card, 49 x 34 cm, illustrative of a Japanese visual acuity chart produced for promotional purposes by the ... The last column has Snellen decimal visual acuity notation. The background has decorative bamboo tree and leaves and ...
The phrase "visual acuity" refers to the clarity of your vision. Experts consider normal, or healthy, visual acuity to be 20/20 ... Visual acuity: What is 20/20 vision? (n.d.).. aoa.org/patients-and-public/eye-and-vision-problems/glossary-of-eye-and-vision- ... In that situation, the only real limit to how far you can clearly see is your eye health and visual acuity. ... Aside from obvious visual obstructions like trees, buildings, and clouds, theres one major factor that can reduce your ...
Synonyms for visual acuity - visual acuity, acuity, and others. ... this page you will find all the synonyms for the word to visual ... Synonyms for acuity. visual acuity (sharpness, , >>). acuteness. See all synonyms for acuity ... visual acuity. Synonyms for visual. graphical. graphic. visible. optical (optic, ocular, vision >>). See all synonyms for ...
Visual Acuity after Retinal Gene Therapy for Choroideremia. Edwards TL., Jolly JK., Groppe M., Barnard AR., Cottriall CL., ...
... Category: #health By Pankaj Singh Date: 2019-08-05 .soci ... The inclusion criteria had a best corrected visual acuity that was in between 20/40 and 20/200, medium dAMD, retinal pigment ... Also, patients who were given risuteganib met the most primary endpoint gain of 8 letter in visual acuity. ... has reportedly verified that the drug enhances visual acuity in patients suffering from dry age-related macular degeneration, ...
Visual acuity of keratoconic eyes as a function of RGP contact lens base curves. ... Visual Performance. Contact lenses, spectacles and the use of objective vision assessments to predict subjective satisfaction. ...
Test visual acuity. Examine the eyes for conjunctival or corneal damage and treat appropriately. Immediately consult an ... There are infrequent reports of cranial nerve palsies (e.g., facial palsy, hearing loss, visual disturbances) or peripheral ...
Field method to measure visual acuity  World Health Organization; Expert Committee on Epidemiology of Onchocerciasis; Ganley, ...
Visual acuity. * Begin evaluation of the iris at the pupillary margin and move outward. ...
Testing your Visual Acuity and Eyesight Online.. by Mohammed Kateregga. 0. *. Energy Healing ...
Patients underwent visual acuity testing with either Snellen or Illiterate E-chart acuity methods. Snellen visual acuity was ... The Ebola retinal lesions did not affect visual acuity. Overall, no difference was observed in uncorrected visual acuity ... Snellen visual acuity was ,6/7.5 in 75.6% (97.5% CI 63%-85.7%) of EVD survivors and 75.5% (97.5% CI 59.1%-87.9%) of controls. ... to describe the implications for visual acuity, and to assess for EBOV persistence in survivors with cataracts amenable to ...
... Author: Stingl ... Full-field electroretinography, visual acuity and visual fields in Usher syndrome: a multicentre European study. DSpace ...
Testing of side vision (visual field examination). * Visual acuity Visual acuity. The visual acuity test is used to determine ... Yanoff M, Cameron D. Diseases of the visual system. In: Goldman L, Schafer AI, eds. Goldmans Cecil Medicine . 25th ed. ...
Amblyopia affects both near and distance visual acuity equally.[2] Early detection and treatment improve multiple quality of ... It should be noted that even with the resolution of visual acuity deficit, children with anisometropic amblyopia have worse ...
  • We measured changes in presenting visual acuity concentration indices and decomposed them the using a longitudinal approach. (who.int)
  • VQOL declined with reduced performance on the following tests: binocular high contrast visual acuity (p = 0.0011), high contrast visual acuity of the better eye (p = 0.0012), contrast sensitivity (p = 0.005), binocular low contrast visual acuity (p = 0.0065), and high contrast visual acuity of the worse eye (p = 0.015). (bmj.com)
  • Binocular high contrast visual acuity is a good measure of how uveitis patients perform in real life situations. (bmj.com)
  • Background: Although patients with acute optic neuritis (ON) recover high-contrast visual acuity (HCVA) to 20/40 or better in 95% of affected eyes, patients with a history of ON continue to note subjective abnormalities of vision. (elsevierpure.com)
  • The visual acuity test is used to determine the smallest letters you can read on a standardized chart (Snellen chart) or a card held 20 feet (6 meters) away. (medlineplus.gov)
  • The last column has Snellen decimal visual acuity notation. (aco.org.au)
  • Purpose: To determine the impact of postoperative visual function on the vision-related quality of life (VRQoL) in patients after anatomically successful surgery for macula-off rhegmatogenous retinal detachment (RRD) and to propose a classification to grade the extent of macular detachment using preoperative optical coherence tomography (OCT) scans. (eyehospital.nl)
  • At each visit, patients underwent detailed ophthalmological examination and anatomical and functional outcomes, silicone oil emulsification, intraocular inflammation, presence of proliferative vitreoretinopathy, preoperative and postoperative visual acuity, and postoperative complications were recorded. (bvsalud.org)
  • Visual Acuity Score (VAS) is an inversion of the logMAR scale, based on VAS=100-50xlogMAR. (visualacuity.info)
  • High (monocular and binocular) and low (binocular) contrast logMAR letter acuities were recorded using a Bailey-Lovie chart. (bmj.com)
  • Best-corrected visual acuity (BCVA) was tested with Landolt rings using an automated device (VA-CAL test). (bvsalud.org)
  • Translational Relevance The VA-CAL test discovers losses of spatial resolution in the visual acuity space not seen in standardized BCVA assessment. (bvsalud.org)
  • At 12 months after surgery, visual function assessments were as follows: metamorphopsia (M-CHARTS), aniseikonia (New Aniseikonia Test), best corrected visual acuity (BCVA) (Early Treatment Diabetic Retinopathy Study [ETDRS]), low contrast BCVA (10% ETDRS), color vision (Hardy Rand Rittler), and stereopsis (Titmus Fly). (eyehospital.nl)
  • The mean (standard deviation) for presenting visual acuity and economic status scores in 2009 versus 2014 were 0.090 (0.2) versus 0.103 (0.2) and 0.01 (1.0) versus 0.0005 (1.07), respectively. (who.int)
  • 0.01) to be the only visual function adequate to predict VQOL. (bmj.com)
  • To investigate the correlation between tests of visual function and perceived visual ability recorded with a quality of life questionnaire for patients with uveitis. (bmj.com)
  • Moreover, partial correlations showed a strong correlation between cognition and acuity when controlling for age but no significant correlation between age and acuity when controlling for cognition. (reading.ac.uk)
  • Methods The predictive ability of chronological age and cognition on acuity were assessed in a group of 81 typical children between five and eleven years old. (reading.ac.uk)
  • Methods: Data from an ongoing collaborative study of visual outcomes in multiple sclerosis and ON were analyzed for this cross-sectional observational cohort. (elsevierpure.com)
  • The inclusion criteria had a best corrected visual acuity that was in between 20/40 and 20/200, medium dAMD, retinal pigment epithelial as well as a preserved foveal outer retina. (intelligencejournal.com)
  • La agudeza visual depende de las funciones de la RETINA, de la transmisión neuronal, y de la capacidad interpretativa del cerebro. (bvsalud.org)
  • Visual acuity depends on the functions of RETINA, neuronal transmission, and the interpretative ability of the brain. (bvsalud.org)
  • Amblyopia affects both near and distance visual acuity equally. (medscape.com)
  • It should be noted that even with the resolution of visual acuity deficit, children with anisometropic amblyopia have worse stereoacuity when compared with nonamblyopic children of the same age. (medscape.com)
  • For uncorrected visual acuity, you will be asked to remove your glasses or contact lenses and stand or sit 20 feet (6 meters) from the eye chart. (medlineplus.gov)
  • Testing for myopia takes several procedures and the first step is often to check distant visual acuity through a visual acuity chart. (visualacuity.info)
  • Card, 49 x 34 cm, illustrative of a Japanese visual acuity chart produced for promotional purposes by the Hoya Lens Co. A note on the back states that the chart is for purely decorative purposes and is in the style of a Japanese 'Noren' or sign curtain that is commonly hung over Japanese shop entrances. (aco.org.au)
  • It is reduced in size relative to a visual acuity chart and is not intended to be used for visual acuity testing. (aco.org.au)
  • Visual acuity was measured using Snellens' chart, and the fundus was examined with direct ophthalmoscope. (bvsalud.org)
  • And the decrement of visual acuity may indicate the development of myopia. (visualacuity.info)
  • Designed for children older than 6, parents with young children, and general practitioners, this app displays optically precise optotypes of eye charts for you to measure distant visual acuity accurately in order to detect nearsightedness or myopia early, so you can take preventive measures and seek help from medical professional early to prevent pseudomyopia from becoming myopia, or prevent nearsightedness from getting worse. (visualacuity.info)
  • The visual acuity test is a routine part of an eye examination or general physical examination, particularly if there is a change in vision or a problem with vision. (medlineplus.gov)
  • This girl's examination showed a distance visual acuity of 10/10 in her right eye. (asrjetsjournal.org)
  • We examined the test-retest reliability of visual acuity in 18 children referred to the Hospital Eye Service with reduced vision. (nih.gov)
  • The phrase "visual acuity" refers to the clarity of your vision. (healthline.com)
  • Experts consider normal, or healthy, visual acuity to be 20/20 vision. (healthline.com)
  • Vision quality of life is worst in younger patients with poor binocular visual acuity. (bmj.com)
  • We measured vision-specific quality of life (QOL) in patients with a history of acute ON and recovery of VA to 20/40 or better in their affected eyes to determine how these QOL scores relate to RNFL and GCL thickness and low-contrast letter acuity (LCLA) across the spectrum of visual recovery. (elsevierpure.com)
  • Conclusions: Patients with a history of ON and good visual recovery, defined in the literature as 20/40 or better HCVA, are left with clinically meaningful reductions in vision-specific QOL. (elsevierpure.com)
  • This study considers whether cognition might also be a predictor of acuity in children. (reading.ac.uk)
  • However, these studies almost exclusively use chronological age as the sole predictor for visual development. (reading.ac.uk)
  • Since many of the tasks used to measure acuity have a cognitive demand, it is possible that age is not the best predictor for changes in this function. (reading.ac.uk)
  • The aim of this study was to explore the effect of cognition on the development of visual acuity, and to compare this predictor with age. (reading.ac.uk)
  • Results Analysis of resulting trajectories show that while age indeed was a good predictor, development of visual acuity was equally well predicted by cognition. (reading.ac.uk)
  • Conclusions These results suggest that age alone is not the optimal determinant for the development of visual acuity in typical school age children, as cognition was also found to be an important predictor. (reading.ac.uk)
  • The extent of macular detachment is a potential predictor for visual function and can be graded using the proposed classification. (eyehospital.nl)
  • A further analysis of 14 subjects' data each of whom had one eye with consistently poorer acuity in both test sessions yielded coefficients of repeatability of +/- 0.15 and +/- 0.22 log units in, respectively, the better and poorer eyes. (nih.gov)
  • Results from a Phase 2 clinical trial, which assessed the efficacies of Risuteganib in macular degeneration patients, has reportedly verified that the drug enhances visual acuity in patients suffering from dry age-related macular degeneration, an ailment more commonly known as dAMD. (intelligencejournal.com)
  • The main cause of visual impairment was cystoid macular oedema. (bmj.com)
  • Patients with stage 3 macular detachments had the highest visual function values compared to the other stages. (eyehospital.nl)
  • 20/40 or better visual acuity after optic neuritis: Not as good as we once thought? (elsevierpure.com)
  • Dive into the research topics of '20/40 or better visual acuity after optic neuritis: Not as good as we once thought? (elsevierpure.com)
  • The VA-CAL Test Quantifies Improvement of Visual Acuity in Achromatopsia by Means of Short-Wave Cutoff Filter Glasses in Daily Living Conditions. (bvsalud.org)
  • To quantify visual performance of patients with achromatopsia at various contrast and luminance combinations typical for daily living conditions , in comparison to controls, and to measure beneficial effects of short-wavelength cutoff filter glasses used by patients with achromatopsia to reduce glare sensation . (bvsalud.org)
  • The VA-CAL test provides numerical proof that short-wavelength cutoff filter glasses can help patients with achromatopsia in everyday life , avoiding the common situation of severe visual impairment at certain daily object contrasts and ambient luminances. (bvsalud.org)
  • Filter glasses improve the patients ' daily visual performance, rendering them a strongly recommended visual aid in achromatopsia . (bvsalud.org)
  • Uveitis is a major cause of severe visual impairment. (bmj.com)
  • 1 Bilateral legal blindness developed in 22 (4%) of the patients, 26 (4.5%) had one blind eye with visual impairment of the other, and nine (1.5%) had bilateral visual impairment. (bmj.com)
  • Unilateral losses occurred in 146 (25%), blindness in 82 (14%), and visual impairment in 64 (11%) patients. (bmj.com)
  • 4 In the 1993 annual report of Research to Prevent Blindness, an estimated 2 300 000 Americans suffered visual impairment resulting from uveitis. (bmj.com)
  • As uveitis mostly affects individuals of working age this visual impairment has important economic consequences with numerous days off work or job losses, and in the younger age group interferes with education. (bmj.com)
  • Children's visual impairment is a significant barrier to their education, especially in low-income countries, due to the scarcity of specialized institutions for this purpose. (asrjetsjournal.org)
  • Draft action plan for the prevention of avoidable blindness and visual impairment 2014-2019. (asrjetsjournal.org)
  • Moreover, when DVA is measured, not only the minimum spatial separation that the visual system can resolve is evaluated, but also the functionality of the oculomotor system. (bvsalud.org)
  • Such patient-observed deficits reflect the underlying significant degrees of retinal axonal and neuronal loss and visual dysfunction that are now known to characterize ON even in the setting of maximal HCVA recovery. (elsevierpure.com)
  • Although patients with 20/50 or worse HCVA recovery demonstrated the worst performance on low-contrast acuity, affected eye RNFL and GCL + IPL thickness, and QOL scales, these measures were also significantly reduced among those with 20/40 or better HCVA recovery compared with controls. (elsevierpure.com)
  • Most patients with 20/40 or better visual recovery (98/112, 88%) had monocular HCVA in their affected eye of 20/20 or better. (elsevierpure.com)
  • How is visual acuity for both eyes determined? (aao.org)
  • That will give you your visual acuity for both eyes. (aao.org)
  • But, a sightline is really any uninterrupted visual angle from a person's eyes to what the person is trying to see. (healthline.com)
  • Unaided distance acuities were recorded, using logarithmically scaled letter charts, on two occasions separated by an interval of one week during which no clinical intervention took place. (nih.gov)
  • Reports cite, patients having non-exudative dAMD, when given risuteganib treatment, saw a gain of 8 letter in terms of visual acuity. (intelligencejournal.com)
  • Also, patients who were given risuteganib met the most primary endpoint gain of 8 letter in visual acuity. (intelligencejournal.com)
  • Purpose Prior studies have shown that changes in visual acuity in typically developing children occur beyond primary school age. (reading.ac.uk)
  • To measure and decompose the over-time changes in economic inequality in presenting visual acuity measured using Logarithm of the Minimum Angle of Resolution. (who.int)
  • A 12-year-old Girl with a Distance Visual Acuity of 10/10 (OD) and Light Perception (OS), Found in a School for Blind People in a Town Located in Central Côte d'Ivoire: An Isolated Case? (asrjetsjournal.org)
  • Normal visual acuity is expressed as 20/20 indicating that one can see at 20 feet what should normally be seen at that distance. (bvsalud.org)
  • Kaiser also stated that, this treatment might not work for all dAMD patients, it might only work for the people having problem with visual acuity. (intelligencejournal.com)
  • 1 A report on 582 uveitis patients showed that 203 (35%) suffered from significant visual loss. (bmj.com)
  • Another study on 148 uveitis patients, which examined the incidence, cause, and duration of visual loss, showed that 86 patients (58.1%) had reduced visual acuity in at least one eye at some period during their disease. (bmj.com)
  • Patients and disease-free control participants completed the 25-Item National Eye Institute Visual Functioning Questionnaire (NEI-VFQ-25) and 10-Item Neuro-Ophthalmic Supplement to the NEI-VFQ-25, as well as VA and LCLA testing for each eye separately and binocularly. (elsevierpure.com)
  • As a result tiveness of trabeculectomy in lowering of intra ocular some patients will require surgery to preserve their vi- pressure (IOP) and preservation of visual acuity. (who.int)
  • The reliability of visual acuity recorded in the better eye closely approximates to that previously found in normal adults, whilst that of the poorer eye appears more comparable to that of adult clinical populations. (nih.gov)
  • We analysed 4706 participants who had complete data on presenting visual acuity and economic status in 2009 and 2014 in the Shahroud Eye Cohort Study. (who.int)
  • Unexpectedly, reduction in economic inequality in presenting visual acuity was due to presenting visual acuity deterioration among the higher economic status group rather than its amelioration among the lower economic status group. (who.int)
  • Therefore, the needs of all socioeconomic groups should be considered separately to modify presenting visual acuity in each group and, consequently, reduce the economic inequality in presenting visual acuity. (who.int)
  • Yet the effects of early-life exposure on visual and auditory function are unknown. (cdc.gov)
  • Significance The development of visual acuity has often been looked upon as a function of age. (reading.ac.uk)
  • Associations between visual function parameters and NEIVFQ-25 scores were evaluated. (eyehospital.nl)
  • We present a review on the visual ability to discriminate fine details of moving objects (DVA: Dynamic Visual Acuity), showing the most relevant differences, which have been attributed to this visual capacity in comparison to SVA (static visual acuity). (bvsalud.org)
  • We used the data collected in a cohort study and applied a dynamic decomposition method (8) to analyse the longitudinal changes in economic inequality in presenting visual acuity (PVA), as one of the health outcomes that have a strong and fully proved relationship with socioeconomic status (7,9-14). (who.int)
  • Our study aimed to detect if any specific retinal signs can be attributed to past EVD in survivors, to describe the implications for visual acuity, and to assess for EBOV persistence in survivors with cataracts amenable to cataract surgery where no intraocular inflammation was present. (cdc.gov)
  • Approval for the study was obtained from the Research of glaucoma, eye operated , pre-operative and post- and Ethics Committee of the Obafemi Awolowo Uni- operative visual acuity, intra-ocular pressure at presen- versity Teaching Hospital Complex, Osun State, Nige- tation , pre-operatively and post-operatively ( day 1, ria. (who.int)
  • Through enabling "Record Scores" in Settings, you can keep track of the progression of your visual acuity. (visualacuity.info)
  • VRQoL was assessed by the National Eye Institute Visual Functioning Questionnaire-25 (NEIVFQ-25). (eyehospital.nl)
  • However, little is known regarding the medium- to long-term visual outcome of survivors or the rates of background uveitis and chorioretinal lesions within the local population. (cdc.gov)
  • La agudeza visual normal se expresa como 20 de 20, que indica que se puede ver a 20 pies lo que normalmente debería ser visto a esa distancia. (bvsalud.org)
  • Please visit optometrists regularly and seek help from medical professional at early signs of visual acuity problems. (visualacuity.info)
  • Longitudinal decomposition of this change in concentration indices during the 5-year period indicated that the most important contributor to reduction in economic inequality of presenting visual acuity was deterioration of presenting visual acuity among people with higher economic status due to their ageing. (who.int)
  • However, the disruption of visual or auditory pathway maturation in infancy could potentially negatively affect downstream cognitive development. (cdc.gov)