Digestive System and Oral Physiological Phenomena
Reproductive and Urinary Physiological Phenomena
Musculoskeletal and Neural Physiological Phenomena
Circulatory and Respiratory Physiological Phenomena
Integumentary System Physiological Phenomena
Sports Nutritional Physiological Phenomena
Reproductive Physiological Phenomena
Elder Nutritional Physiological Phenomena
Urinary Tract Physiological Phenomena
Digestive System Physiological Phenomena
Simian virus 40
Influenza A Virus, H1N1 Subtype
Influenza A Virus, H5N1 Subtype
Ocular Physiological Phenomena
Influenza A Virus, H3N2 Subtype
Hepatitis B virus
West Nile virus
Nervous System Physiological Phenomena
Respiratory Syncytial Viruses
Respiratory Physiological Phenomena
Skin Physiological Phenomena
Plant Physiological Phenomena
Mechanisms of viral interference with MHC class I antigen processing and presentation. (1/191)Viruses are ubiquitous and dangerous obligate intracellular parasites. To facilitate recognition of virus-infected cells by the immune system, vertebrates evolved a system that displays oligopeptides derived from viral proteins on the surface of cells in association with class I molecules of the major histocompatibility complex. Here we review the mechanisms counter-evolved by viruses to interfere with the generation of viral peptides, their intracellular trafficking, or the cell surface expression of class I molecules bearing viral peptides. This topic is important in its own right because the viruses that encode these proteins represent medically important pathogens, are potential vectors for vaccines or gene therapy, and provide strategies and tools for blocking immune recognition in transplantation, autoimmunity, and gene therapy. In addition, studies on viral interference provide unique insights into unfettered antigen processing and normal cellular functions that are exploited and exaggerated by viruses. (+info)
Virioplankton: viruses in aquatic ecosystems. (2/191)The discovery that viruses may be the most abundant organisms in natural waters, surpassing the number of bacteria by an order of magnitude, has inspired a resurgence of interest in viruses in the aquatic environment. Surprisingly little was known of the interaction of viruses and their hosts in nature. In the decade since the reports of extraordinarily large virus populations were published, enumeration of viruses in aquatic environments has demonstrated that the virioplankton are dynamic components of the plankton, changing dramatically in number with geographical location and season. The evidence to date suggests that virioplankton communities are composed principally of bacteriophages and, to a lesser extent, eukaryotic algal viruses. The influence of viral infection and lysis on bacterial and phytoplankton host communities was measurable after new methods were developed and prior knowledge of bacteriophage biology was incorporated into concepts of parasite and host community interactions. The new methods have yielded data showing that viral infection can have a significant impact on bacteria and unicellular algae populations and supporting the hypothesis that viruses play a significant role in microbial food webs. Besides predation limiting bacteria and phytoplankton populations, the specific nature of virus-host interaction raises the intriguing possibility that viral infection influences the structure and diversity of aquatic microbial communities. Novel applications of molecular genetic techniques have provided good evidence that viral infection can significantly influence the composition and diversity of aquatic microbial communities. (+info)
Viruses at the edge of adaptation. (3/191)How vulnerable is the line that separates adaptation from extinction? Viruses, in particular RNA viruses, are well known for their high rates of genetic variation and their potential to adapt to environmental modifications (Drake and Holland, 1999; Domingo et al., 2000). Yet, fitness variations-both increases and decreases-can be spectacularly rapid, and the simple genetic stratagem of forcing virus multiplication to go through repeated genetic bottlenecks can induce fitness losses, at times near viral extinction. New information has been recently obtained on the two sides of the survival line: the edge of adaptation and the edge of extinction. (+info)
Identification of distinct signaling pathways leading to the phosphorylation of interferon regulatory factor 3. (4/191)Infection of host cells by viruses leads to the activation of multiple signaling pathways, resulting in the expression of host genes involved in the establishment of the antiviral state. Among the transcription factors mediating the immediate response to virus is interferon regulatory factor-3 (IRF-3) which is post-translationally modified as a result of virus infection. Phosphorylation of latent cytoplasmic IRF-3 on serine and threonine residues in the C-terminal region leads to dimerization, cytoplasmic to nuclear translocation, association with the p300/CBP coactivator, and stimulation of DNA binding and transcriptional activities. We now demonstrate that IRF-3 is a phosphoprotein that is uniquely activated via virus-dependent C-terminal phosphorylation. Paramyxoviridae including measles virus and rhabdoviridae, vesicular stomatitis virus, are potent inducers of a unique virus-activated kinase activity. In contrast, stress inducers, growth factors, DNA-damaging agents, and cytokines do not induce C-terminal IRF-3 phosphorylation, translocation or transactivation, but rather activate a MAPKKK-related signaling pathway that results in N-terminal IRF-3 phosphorylation. The failure of numerous well characterized pharmacological inhibitors to abrogate virus-induced IRF-3 phosphorylation suggests the involvement of a novel kinase activity in IRF-3 regulation by viruses. (+info)
Osteoclasts and giant cells: macrophage-macrophage fusion mechanism. (5/191)Membrane fusion is a ubiquitous event that occurs in a wide range of biological processes. While intracellular membrane fusion mediating organelle trafficking is well understood, much less is known about cell-cell fusion mediating sperm cell-oocyte, myoblast-myoblast and macrophage-macrophage fusion. In the case of mononuclear phagocytes, their fusion is not only associated with the differentiation of osteoclasts, cells which play a key role in the pathogenesis of osteoporosis, but also of giant cells that are present in chronic inflammatory reactions and in tumours. Despite the biological and pathophysiological importance of intercellular fusion events, the actual molecular mechanism of macrophage fusion is still unclear. One of the main research themes in my laboratory has been to investigate the molecular mechanism of mononuclear phagocyte fusion. Our hypothesis has been that macrophage-macrophage fusion, similar to virus-cell fusion, is mediated by specific cell surface proteins. But, in contrast with myoblasts and sperm cells, macrophage fusion is a rare event that occurs in specific instances. To test our hypothesis, we established an in vitro cell-cell fusion assay as a model system which uses alveolar macrophages. Upon multinucleation, these macrophages acquire the osteoclast phenotype. This indicates that multinucleation of macrophages leads to a specific and novel functional phenotype in macrophages. To identify the components of the fusion machinery, we generated four monoclonal antibodies (mAbs) which block the fusion of alveolar macrophages and purified the unique antigen recognized by these mAbs. This led us to the cloning of MFR (Macrophage Fusion Receptor). MFR was cloned simultaneously as P84/SHPS-1/SIRPalpha/BIT by other laboratories. We subsequently showed that the recombinant extracellular domain of MFR blocks fusion. Most recently, we identified a lower molecular weight form of MFR that is missing two extracellular immunoglobulin (Ig) C domains. Shortly after we cloned MFR, CD47 was reported to be a ligand for P84/SIRPalpha. We have since generated preliminary results which suggest that CD47 interacts with MFR during adhesion/fusion and is a member of the fusion machinery. We also identified CD44 as a plasma membrane protein which, like MFR, is highly expressed at the onset of fusion. The recombinant soluble extracellular domain of CD44 blocks fusion by interacting with a cell-surface binding site. We now propose a model in which both forms of MFR, CD44, and CD47 mediate macrophage adhesion/fusion and therefore the differentiation of osteoclasts and giant cells. (+info)
Efficient oncolysis by a replicating adenovirus (ad) in vivo is critically dependent on tumor expression of primary ad receptors. (6/191)Replicating adenoviruses (Ads) are designed to replicate in and destroy cancer cells, generating viral progeny that spread within the tumor. To address the importance of the primary cellular receptor for Ads, the coxsackievirus and Ad receptor (CAR), in permitting intratumoral spread of a replicating Ad, we have used a pair of tumor cell lines differing only in the expression of a primary receptor for Ad5. This novel system thus allowed the first direct evaluation of the relationship between the efficacy of a replicating Ad and the primary receptor levels of the host cell without the confounding influence of other variable cellular factors. We demonstrate that the absence of the primary cellular receptor on the tumor cells restricts the oncolytic potency of a replicating Ad both in vitro and in vivo. Based on these findings, it is apparent that the potential therapeutic advantages afforded by viral replication would be negated by poor intratumoral spread of the viral progeny due to the failure to infect neighboring tumor cells. Because a number of studies have reported that primary cancer cells express only low levels of CAR, our results suggest that strategies to redirect Ads to achieve CAR-independent infection will be necessary to realize the full potential of replicating Ads in the clinical setting. (+info)
The dependence of viral parameter estimates on the assumed viral life cycle: limitations of studies of viral load data. (7/191)Estimation of viral parameters, such as the basic reproductive number (R0) and infected cell life span, is central to the quantitative study of the within-host dynamics of viral diseases such as human immunodeficiency virus, hepatitis B or hepatitis C. As these parameters can rarely be determined directly, they are usually estimated indirectly by fitting mathematical models to viral load data. This paper investigates how parameter estimates obtained by such procedures depend on the assumptions made concerning the viral life cycle. It finds that estimates of the basic reproductive number obtained using viral load data collected during the initial stages of infection can depend quite sensitively on these assumptions. The use of models which neglect the intracellular delay before virion production can lead to severe underestimates of R0 and, hence, to overly optimistic predictions of how efficacious treatment must be in order to prevent or eradicate the disease. These results are also of importance for attempts at estimating R0 from similar epidemiological data as there is a correspondence between within-host and between-host models. Estimates of the life span of infected cells obtained from viral load data collected during drug treatment studies also depend on the assumptions made in modelling the virus life cycle. The use of more realistic descriptions of the life cycle is seen to increase estimates of infected cell life span, in addition to providing a new explanation for the shoulder phase seen during drug treatment. This study highlights the limitations of what can be learnt by fitting mathematical models to infectious disease data without detailed independent knowledge of the life cycle of the infectious agent. (+info)
Lung surfactant and reactive oxygen-nitrogen species: antimicrobial activity and host-pathogen interactions. (8/191)Surfactant protein (SP) A and SP-D are members of the collectin superfamily. They are widely distributed within the lung, are capable of antigen recognition, and can discern self versus nonself. SPs recognize bacteria, fungi, and viruses by binding mannose and N-acetylglucosamine residues on microbial cell walls. SP-A has been shown to stimulate the respiratory burst as well as nitric oxide synthase expression by alveolar macrophages. Although nitric oxide (NO.) is a well-recognized microbicidal product of macrophages, the mechanism(s) by which NO. contributes to host defense remains undefined. The purpose of this symposium was to present current research pertaining to the specific role of SPs and reactive oxygen-nitrogen species in innate immunity. The symposium focused on the mechanisms of NO*-mediated toxicity for bacterial, human, and animal models of SP-A- and NO.-mediated pathogen killing, microbial defense mechanisms against reactive oxygen-nitrogen species, specific examples and signaling pathways involved in the SP-A-mediated killing of pulmonary pathogens, the structure and binding of SP-A and SP-D to bacterial targets, and the immunoregulatory functions of SP-A. (+info)
1. Common cold: A viral infection that affects the upper respiratory tract and causes symptoms such as sneezing, running nose, coughing, and mild fever.
2. Influenza (flu): A viral infection that can cause severe respiratory illness, including pneumonia, bronchitis, and sinus and ear infections.
3. Measles: A highly contagious viral infection that causes fever, rashes, coughing, and redness of the eyes.
4. Rubella (German measles): A mild viral infection that can cause fever, rashes, headache, and swollen lymph nodes.
5. Chickenpox: A highly contagious viral infection that causes fever, itching, and a characteristic rash of small blisters on the skin.
6. Herpes simplex virus (HSV): A viral infection that can cause genital herpes, cold sores, or other skin lesions.
7. Human immunodeficiency virus (HIV): A viral infection that attacks the immune system and can lead to acquired immunodeficiency syndrome (AIDS).
8. Hepatitis B: A viral infection that affects the liver, causing inflammation and damage to liver cells.
9. Hepatitis C: Another viral infection that affects the liver, often leading to chronic liver disease and liver cancer.
10. Ebola: A deadly viral infection that causes fever, vomiting, diarrhea, and internal bleeding.
11. SARS (severe acute respiratory syndrome): A viral infection that can cause severe respiratory illness, including pneumonia and respiratory failure.
12. West Nile virus: A viral infection that can cause fever, headache, and muscle pain, as well as more severe symptoms such as meningitis or encephalitis.
Viral infections can be spread through contact with an infected person or contaminated surfaces, objects, or insects such as mosquitoes. Prevention strategies include:
1. Practicing good hygiene, such as washing hands frequently and thoroughly.
2. Avoiding close contact with people who are sick.
3. Covering the mouth and nose when coughing or sneezing.
4. Avoiding sharing personal items such as towels or utensils.
5. Using condoms or other barrier methods during sexual activity.
6. Getting vaccinated against certain viral infections, such as HPV and hepatitis B.
7. Using insect repellents to prevent mosquito bites.
8. Screening blood products and organs for certain viruses before transfusion or transplantation.
Treatment for viral infections depends on the specific virus and the severity of the illness. Antiviral medications may be used to reduce the replication of the virus and alleviate symptoms. In severe cases, hospitalization may be necessary to provide supportive care such as intravenous fluids, oxygen therapy, or mechanical ventilation.
Prevention is key in avoiding viral infections, so taking the necessary precautions and practicing good hygiene can go a long way in protecting oneself and others from these common and potentially debilitating illnesses.
Outline of biology
List of MeSH codes
Japanese tree frog
Plant-induced systemic resistance
Preben von Magnus
Balo concentric sclerosis
Nipple pain in breastfeeding
ESA Scientific Research on the International Space Station
Occupational safety and health
Conditioned taste aversion
Lipid bilayer fusion
Risk factors of schizophrenia
Endothelial cell tropism
Alice in Wonderland syndrome
De novo gene birth
Medical students' disease
Cancer signs and symptoms
Dawkins vs. Gould
Virus Latency | Harvard Catalyst Profiles | Harvard Catalyst
Temporal Stability of the Human Skin Microbiome - PubMed
Portal Regional da BVS
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iCite | User Guide | NIH Office of Portfolio Analysis
Differential Virulence of West Nile Strains for American Crows - Volume 10, Number 12-December 2004 - Emerging Infectious...
Liver Congestion Causes Chronic Fatigue - GRIN
Tumor virus infections. Medical search. Definitions
Virus Inactivation | Profiles RNS
Geförderte Projekte für Postdocs (ZF 04) - Universität Bremen
September 2020 - OVDoll: Best Real Lifesize Silicone and TPE Sex Dolls: Best Adult Lifelike Cheap Realistic Love Dolls Shop
PA-18-738: Age-related Microbiota Changes and their Implications in Chronic Disease Prevention, Treatment and Progression (R01...
SMART: DISIN domain annotation
NIH Director's Transformative Research Award Program - 2009 Award Recipients | NIH Common Fund
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Retroviruses: A broad view of SARS-COV-2 and its relatives, with a narrative essay on the current state of biomedical sciences ...
Xuehua Xu, Ph.D. | NIH: National Institute of Allergy and Infectious Diseases
- By varying host conditions (permissivity to viral entry T and immune clearance intensity A ) for large numbers of cells and viruses, we study the dynamics of how viral populations evolve from initial infection to steady state and obtain a phase diagram of the range of cell and viral responses. (biorxiv.org)
- We find three distinct replicative strategies corresponding to three physiological classes of viral infections: acute, chronic, and opportunistic. (biorxiv.org)
- In our model, the viruses encounter host cell defenses characterized by two parameters: permissivity to viral entry T and immune response A ). The viruses then mutate upon reproduction, eventually resulting in a distribution of related viral types termed a quasi-species distribution. (biorxiv.org)
- Analysis of viral of viral mutant populations over a wide range of permissivity and immunity, for large numbers of cells and viruses, reveals phase transitions that separate the three classes of viruses, both in the infection-cycle dynamics and at steady state. (biorxiv.org)
- The presence of the virus in the bloodstream, its ability to directly penetrate the NS through peripheral nerves and the weakening of the blood-brain barrier are added to the tropism of SARS-CoV-2 by ACE-2 receptors, which favours the appearance of brain manifestations, associated with metabolic complications in the autoimmune processes induced by viral clinical condition. (itmedicalteam.pl)
Normal physiological conditions2
- At some point, their functions are diminished whereas favorable or rather normal physiological conditions enhance normal functioning. (grin.com)
- Under normal physiological conditions, when the acidic contents of the stomach and food fats enter the duodenum, the sphincter of Oddi opens, a moderate contraction of the gallbladder and the release of bile into the intestine occur. (pastaplusrestaurant.com)
- Either the virus is complexed with antiviral IMMUNOGLOBULIN G and binds to Fc receptors, or virus is coated with antiviral IMMUNOGLOBULIN M and binds to complement receptors. (childrensmercy.org)
- Biological properties, processes, and activities of VIRUSES , including the interactions with the cells they infect. (nih.gov)
- Ideally, an individual's body is said to be normal and healthy when all biological processes and organs are functioning normally under optimal physiological conditions. (grin.com)
- This cellular behavior of eukaryotic cells plays a critical role in many physiological processes, such as embryogenesis, neuron patterning, angiogenesis, innate immune responses to infections, metastasis of cancer cells, and the early development of the model organism Dictyostelium . (nih.gov)
- Dr. Xu has long-term research interests in GPCR functions in certain fundamental processes, such as human immunodeficiency virus (HIV) infections and phagocytosis of pathogens, with the ultimate goal of revealing novel therapeutic strategies for inflammation-related diseases. (nih.gov)
- It's clear that the human organism has two defense strategies, and that the immune system and physiological system should be understood in a more integrated manner when analyzing infectious processes," Steiner told. (eurekalert.org)
- A class of cysteine proteases which play an essential role in VIRUS REPLICATION. (nih.gov)
- In our model, viruses exhibit variable binding to cells, with better infection and replication countered by a stronger immune response and a high rate of mutation. (biorxiv.org)
- The protein affects=20 replication of the Rubella virus RNA [35,36]. (bio.net)
- Both Dictyostelium and neutrophils sense chemoattractant gradients over a huge concentration range through a cellular phenomenon known as adaptation. (nih.gov)
- I've been critically examining the kind of distortions that I have had to engage due to the phenomenon of 'societal' adaptation and I have come to view the confusion regarding 'adaptation being an element of evolution' as one of the many subtle distortions of perspective that the social virus 1 likes to utilise. (songsfortheotherkind.com)
- Specific molecular components of the cell capable of recognizing and interacting with a virus, and which, after binding it, are capable of generating some signal that initiates the chain of events leading to the biological response. (lookformedical.com)
- A closely related virus that circulates in Australia (Kunjin [KUN]) has never been associated with outbreaks of human or animal diseases, including bird diseases, nor have bird deaths been reported from enzootic transmission foci in Africa, where a virus that shares 96.5% nucleotide identity with the NY99 strain has previously been isolated ( 16 , 17 ). (cdc.gov)
- Virus diseases caused by the RETROVIRIDAE . (lookformedical.com)
- A general term for diseases produced by viruses. (lookformedical.com)
- Virus diseases caused by the ORTHOMYXOVIRIDAE . (lookformedical.com)
- In most cases, diseases are caused by microorganisms such as bacteria, viruses and fungus. (grin.com)
- Diagnosis and Epidemiology of plant viruses and other diseases. (moa.gov.cy)
- Of particular interest to our group are the mechanisms by which Bcl-2 family proteins and other factors regulate programmed cell death , particularly in the nervous system, in cancer and in virus infections. (hopkinsmedicine.org)
- We have reported that many insults can trigger cells to activate a cellular death pathway (Nature, 361:739-742, 1993), that several viruses encode proteins to block attempted cell suicide (Proc. (hopkinsmedicine.org)
- In addition, Bcl-2 family proteins have normal physiological roles in regulating mitochondrial fission/fusion and mitochondrial energetics to facilitate neuronal activity in healthy brains. (hopkinsmedicine.org)
- Proteins found in any species of virus. (lookformedical.com)
- A subtype of INFLUENZA A VIRUS with the surface proteins hemagglutinin 1 and neuraminidase 1. (lookformedical.com)
- Viruses parasitic on plants higher than bacteria. (lookformedical.com)
- A virus capsid-like nanocompartment that stores iron and protects bacteria from oxidative stress. (nih.gov)
- The natural process which leads to the cell death is called as apoptosis , but when the cell death happens due to attack certain external factors like bacteria, virus or fungus, or any toxicity, injury, leads to necrosis . (biodifferences.com)
- Meanwhile, research in the field of immunology highlighted the phenomenon of immune system activation with the primary aim of killing a pathogen. (eurekalert.org)
- There are many morphological, physiological and biochemical differences in both the process. (biodifferences.com)
- 94: 690-694, 1997), that cellular anti-death genes can alter the pathogenesis of virus infections (Nature Med. (hopkinsmedicine.org)
- Tumor virus infections. (lookformedical.com)
- Infections produced by oncogenic viruses . (lookformedical.com)
- The infections caused by DNA viruses are less numerous but more diverse than those caused by the RNA oncogenic viruses . (lookformedical.com)
- The Ebolavirus genus includes 5 different viruses that result in different case-fatality rates: Ebola virus, Sudan virus, and Bundibugyo virus cause fatal infections, but neither Tai Forest virus nor Reston virus has been associated with human fatalities. (cdc.gov)
- For example, nosocomial Ebola virus disease infections mostly occur in adults working on hospital wards, and children are not usually caregivers for Ebola virus disease patients. (cdc.gov)
- while on the surface the individual is scrambling to adjust, be it physically, emotionally, psychologically, economically or a combination of any of these, the real damage is being done on the deepest psychological and physiological levels, which is precisely the desired effect. (songsfortheotherkind.com)
- I would like to reflect these definitions in light of the effect that a dominating element- the Virus- causes by stealth to be absorbed into the psychological landscape of an entire culture: it is by these psychological and linguistic sleights of hand that the trick of stealing an individual's autonomy is effected. (songsfortheotherkind.com)
- PCD is an active, orderly cell death process controlled by genes that involves a unique and complex signaling system and it is a normal physiological reaction of the organism in response to stress and its own metabolism (Kerr et al. (researchsquare.com)
- Our simulations also reveal a wide range of physical phenomena, including metastable states, periodicity, and glassy dynamics. (biorxiv.org)
- Physiological data reveal that few nanocompartments are assembled during vegetative growth, but they increase fivefold upon starvation, protecting cells from oxidative stress through iron sequestration. (nih.gov)
- The activation of the coagulation cascade and deregulation of physiological anticoagulant mechanisms, such as protein C system and the disintegration of fibrin, are possible causes of this hypercoagulable state present in COVID-19. (itmedicalteam.pl)
- Process of growing viruses in live animals, plants, or cultured cells. (lookformedical.com)
- From a physiological perspective, excessive intake of noxious substances in the blood circulation causes physiological imbalances of various components including PH and nutrients availability for the cells. (grin.com)
- In theory, the functioning of all body organs depends on the physiological conditions in the body. (grin.com)
- It is known that fiber type, yarn properties, fabric structure, finishing treatments and clothing conditions are the main factors affecting thermo-physiological comfort ( Li, 2001 ). (scialert.net)
- Apoptosis is the self-controlled event that occurs under favourable physiological conditions and the cell itself actively participate in the process. (biodifferences.com)
- Necrosis occurs due to the external like virus, fungi or any toxins. (biodifferences.com)
- In addition, birds that survived challenge with the KEN or KUN viruses were challenged with a lethal dose of the NY99 strain to assess development of a cross-protective immunologic response. (cdc.gov)
- The protein is of high physiological=20 importance as it knockout is embryonic lethal . (bio.net)
- Inactivation of viruses by non-immune related techniques. (ctsicn.org)
- In this regard, it is important to highlight that, besides the direct effect on neurons, the inflammatory state caused by a dysregulation of the immune system due to infection are both factors responsible for neurological phenomena of the disease. (itmedicalteam.pl)
- Substances elaborated by viruses that have antigenic activity. (lookformedical.com)
- The inhibition of TNF-alpha release (and many other shedding phenomena) by hydroxamic acid-based inhibitors indicates that one or more metalloproteinases is involved. (embl.de)
- The ability of a pathogenic virus to lie dormant within a cell (latent infection). (harvard.edu)
- In addition to the samples from the 55 pediatric patients, people less than 21 years of age, who had laboratory-confirmed Ebola virus disease, we selected samples from 50 adult patients, more than 21 years of age, who had laboratory-confirmed infection. (cdc.gov)
- Virus Latency" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, MeSH (Medical Subject Headings) . (harvard.edu)
- Virus Inactivation" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, MeSH (Medical Subject Headings) . (ctsicn.org)
- Pediatric patients have been underrepresented in Ebola virus disease studies because total numbers of affected children in any given Ebola virus disease outbreak, whether associated with Ebola virus or Sudan virus or Bundibugyo virus are usually low because of outbreak dynamics and societal structure. (cdc.gov)
- For the purposes of this discussion some clarification and exploration will be useful: all these definitions relate to the specific element of Virus activity of intentional societal control for an inherent purpose that I'm exploring in this writing. (songsfortheotherkind.com)
- In addition to this, basic properties expected from socks during usage are resistance against abrasion, elasticity, constant post-washing dimensions, thermo-physiological properties and physiological comfort. (scialert.net)
- Author summary Virus, in particular RNA viruses, often produce offspring with slightly altered genetic composition. (biorxiv.org)
- Viruses that produce tumors. (lookformedical.com)
- Paxlovid has better efficacy, but it will be very surprising indeed if treatment via a single mode of action does not eventually produce resistant viruses. (chemistryworld.com)
- Identification of lactate dehydrogenase-elevating virus as the etiological agent of genetically restricted, age-dependent polioencephalomyelitis of mice. (nih.gov)
- Impact of Tamoxifen on Vorinostat-Induced Human Immunodeficiency Virus Expression in Women on Antiretroviral Therapy: AIDS Clinical Trials Group A5366, The MOXIE Trial. (harvard.edu)
- During the 2000-2001 Gulu Ebola virus disease outbreak, an international response team, including representatives from the US Centers for Disease Control and Prevention, or CDC, provided clinical and technical assistance. (cdc.gov)
- On the other hand, deviation from the normal state may be caused by physiological imbalances or deficiencies of essential body requirements, and this is the phenomenon associated with liver congestion. (grin.com)
- Here we try to acquire a "detached" view to some evolutionary and physiological aspects of the human-virus interaction highlighting the need to revitalize science by a strong departure from ultra-specialization toward a real integration of different fields of investigation. (iss.it)
- Virus shedding is an important means of vertical transmission (INFECTIOUS DISEASE TRANSMISSION, VERTICAL). (lookformedical.com)