Ventricular Premature Complexes
Atrial Premature Complexes
Electrocardiography, Ambulatory
Arrhythmias, Cardiac
Electrocardiography
Purkinje Fibers
Heart Failure
Encyclopedias as Topic
Heart Valve Diseases
Cardiomyopathy, Dilated
Heart Valves
Cardiomyopathy, Hypertrophic
Detection of abnormal high-frequency components in the QRS complex by the wavelet transform in patients with idiopathic dilated cardiomyopathy. (1/286)
In order to investigate whether increased fine, fractionated signals within the QRS complex can detect arrhythmogenic substrates and how these fine signals link with ventricular mechanical dysfunction, wavelet analysis was performed on averaged QRS complexes obtained from the left precordial lead in 26 patients with idiopatic dilated cardiomyopathy (IDCM) and in 12 normal subjects. The number of local maxima and the duration of the wavelet transform were significantly greater in patients with IDCM than in normal subjects; the number at 100 Hz was 8.8+/-3.1 vs 6.0+/-1.1 (p<0.01), and the duration at 100Hz was 93+/-15 vs 75+/-7ms (p<0.01). Both of these indices were greater in the patients with than in those without late potentials, repetitive ventricular premature beats or cardiac death. In addition, significant inverse curvilinear relationships were observed between the left ventricular ejection fraction and both the number of local maxima and the duration of the wavelet transform. In conclusion, fine fragmented signals in the QRS complex detected by wavelet analysis would be an important marker for potentially arrhythmogenic substrates and seemed to progress in parallel with left ventricular mechanical dysfunction in IDCM. (+info)Correlation of ventricular mechanosensory neurite activity with myocardial sensory field deformation. (2/286)
The mechanosensory activity generated by ventricular epicardial sensory neurites associated with afferent axons in thoracic sympathetic nerves was correlated with sensory field deformation (long axis, short axis, and transmural dimension changes), regional intramyocardial pressure, and ventricular chamber pressure in anesthetized dogs. Ventricular mechanosensory neurites generated activity that correlated best with strain developed along either the long or short axis of their epicardial sensory fields in most instances. Activity did not correlate normally to local wall thickness or to regional wall or chamber pressure development in most cases. During premature ventricular contractions, the activity generated by these sensory neurites correlated best with maximum strain developed along at least one sensory field epicardial vector. Identified sensory neurites were also activated by local application of the chemical bradykinin (10 microM) or by local ischemia. These data indicate that the activity generated by most ischemia-sensitive ventricular epicardial sensory neurites associated with afferent axons in sympathetic nerves is dependent on not only their local chemical milieu but on local mechanical deformation along at least one epicardial vector of their sensory fields. (+info)Possible theophylline toxicity during anesthesia. (3/286)
Asthmatic patients who undergo outpatient anesthesia are typically prescribed one or more drugs for treatment. Some of these agents have narrow therapeutic ranges and are associated with potentially serious adverse reactions, toxic effects, or drug interactions. Various clinical signs of toxicity may be first uncovered during routine monitoring of an office anesthetic. The case reported here demonstrates the need for proper understanding of the asthmatic patient's medical history and an appreciation for the medications used to control the disease. A sudden cardiovascular event possibly related to drug toxicity is witnessed and treated in an asthmatic patient during intravenous sedation. A possible drug interaction with a non-asthmatic medication taken concomitantly by the patient is implicated and discussed. In addition to the case report, the broad classification of drugs employed for bronchial asthma and their effects is reviewed. (+info)Dental anesthetic management of a patient with ventricular arrhythmias. (4/286)
During routine deep sedation for endodontic therapy, a dentist-anesthesiologist observed premature ventricular contractions (PVCs) on a 62-yr-old woman's electrocardiogram (EKG) tracing. The dentist was able to complete the root canal procedure under intravenous (i.v.) sedation without any problems. The dentist-anesthesiologist referred the patient for medical evaluation. She was found to be free from ischemic cardiac disease with normal ventricular function. The patient was cleared to continue her dental treatment with deep sedation. She subsequently continued to undergo dental treatment with deep intravenous sedation without incident, although her EKG exhibited frequent PVCs, up to 20 per minute, including couplets and episodes of trigeminy. This article will review indications for medical intervention, antiarrhythmic medications, and anesthetic interventions for perioperative PVCs. (+info)Unsuitability of corrected QT dispersion as a marker for ventricular arrhythmias and cardiac sudden death after acute myocardial infarction. (5/286)
The present study investigated whether corrected QT (QTc) dispersion could play a role as a marker of ventricular arrhythmias and sudden cardiac death after acute myocardial infarction (MI). The study included 76 males and 24 females with a mean age of 60+/-11 years. Standard 12-lead ECGs were recorded during the recovery phase (15+/-9 days) after the onset of MI. The QTc was calculated according to Bazett's formula and QTc dispersion was calculated as the difference between the maximum and minimum QTc intervals. Patients were divided into 2 groups: 21 patients (group A) had a QTc dispersion of > or =80ms, and the other 79 patients (group B) had a QTc dispersion of <80ms in the recovery stage (15+/-9 days). Clinical, angiographical, and Holter monitoring data, and prognosis (mean follow-up period 29+/-18 months) were compared between these 2 groups. The frequencies of early coronary reperfusion and recanalization of infarct-related vessels during the recovery phase were significantly higher in group B than group A. The left ventricular ejection fraction was also higher in group B than group A (51+/-12 vs 43+/-12%, p=0.0029). There were no significant differences in the number of premature ventricular contractions, the percentage of patients with repetitive ventricular arrhythmias, or in the frequency of sudden cardiac death during the follow-up period between the 2 groups. In summary, QTc dispersion in the recovery stage is not a useful marker for ventricular arrhythmias or sudden cardiac death after acute MI, although increased QTc dispersion may correlate with an ineffective early coronary reperfusion and with the degree of depressed left ventricular function. (+info)Cyclic bursts of ventricular premature contractions of more than one minute intervals. (6/286)
Ventricular premature contractions (VPCs) occasionally appear successively in the form of bigeminy, trigeminy or quadrigeminy associated with quiescent periods. However, details of these rhythmic VPC bursts have not been well documented. We analyzed the incidence, periodicity and interval of VPC bursts exhibiting bigeminy or trigeminy using ambulatory ECG monitoring and computer analysis. We defined VPC bursts as more than 5 successive groups of VPCs each containing more than 20 VPCs in the form of bigeminy or trigeminy that were interrupted by normal sinus rhythm lasting for more than 60 seconds. Bursts thus defined were observed transiently or continuously in 78 out of 500 consecutive patients showing > 3000 VPCs a day. Their age ranged from 14 to 76 years (mean 48). Forty patients were men and 38 were women. We could discriminate between two types of bursts on the instantaneous heart rate tachograms. Dome type bursts (n = 48) showed gradual shortening of the VPC coupling intervals whereas horizontal type bursts (n = 30) demonstrated fixed coupling intervals during the bursts. Cycle length of the dome type burst was 185 +/- 40 seconds and regular, whereas it was 210 +/- 63 seconds and irregular in the horizontal type (NS). Duration of the VPC bursts was 101 +/- 31 seconds in the dome type and 98 +/- 41 seconds in the horizontal type. Both burst types were associated with transient increases in sinus rate and abbreviated VPC-VPC intervals. We suspect ventricular parasystole to be the mechanism of these bursts especially in the dome type. Recognition of these two burst types from heart rate tachograms may be of value in the suppression of VPCs. (+info)Pacing-induced delayed protection against arrhythmias is attenuated by aminoguanidine, an inhibitor of nitric oxide synthase. (7/286)
1. Cardiac pacing, in anaesthetized dogs, protects against ischaemia and reperfusion-induced ventricular arrhythmias when this is initiated 24 h after the pacing stimulus. Now we have examined whether this delayed cardioprotection afforded by cardiac pacing is mediated through nitric oxide. 2. Twenty-two dogs were paced (4 x 5 min periods at 220 beats min(-1)) by way of the right ventricle, 24 h prior to a 25 min period of coronary artery occlusion. Nine of these dogs were given the inhibitor of induced nitric oxide synthase, aminoguanidine (50 mg kg(-1) i.v.), 0.5 h prior to coronary artery occlusion. Sham-operated non-paced dogs with and without aminoguanidine treatment served as controls. 3. Pacing markedly (P<0. 05) reduced arrhythmia severity (ventricular fibrillation, VF, during occlusion 15%; survival from the combined ischaemia-reperfusion insult 62%) compared to control, sham-operated, unpaced dogs (VF during occlusion 58%; survival 17%). This protection was attenuated by the administration of aminoguanidine prior to coronary artery occlusion (survival from the combined ischaemia-reperfusion insult 11%, which was significantly (P<0.05) less than in the paced dogs not given aminoguanidine and similar to the controls). Aminoguanidine had no significant effects on coronary artery occlusion when given to dogs that had not been paced. In the dose used aminoguanadine transiently elevated systemic arterial pressure by a mean of 20 mmHg and reduced heart rate by a mean of 22 beats min(-1). 4. These results suggest that nitric oxide, probably derived from induced nitric oxide synthase, contributes significantly to the delayed cardioprotection afforded by cardiac pacing. (+info)Heart rate variability analysis of patients with idiopathic left ventricular outflow tract tachycardia: role of triggered activity. (8/286)
There have been several reports with respect to idiopathic ventricular tachycardias (VTs) originating from the left ventricular outflow tract (LVOT). A previous report suggested that triggered activity plays a partial role in idiopathic LVOT tachycardia from the electrophysiological as well as the electropharmacological viewpoint. However, the exact role of triggered activity in this type of VT remains unknown. In the present study the relationship of the frequency of premature ventricular contractions (PVCs) and heart rate was examined and heart rate variability (HRV) was analyzed in 2 cases of LVOT tachycardia using 24-h Holter electrocardiographic (ECG) monitoring. The relation between the PVCs frequency and heart rate showed a persistently positive correlation, indicating frequent PVCs as heart rate increased. In HRV analysis, NN50(%), a time-domain variable of parasympathetic activity, showed no change prior to ventricular arrhythmias. In frequency-domain analysis of HRV, the high frequency (HF) component tended to fall prior to repetitive PVCs and VTs. The ratio of the low frequency to high frequency (LF/HF) components increased prior to single PVCs, repetitive PVCs and VTs. Sympathetic predominance predisposes the genesis of these kinds of arrhythmias originating from the LVOT and it is suggested that triggered activity plays an important role in LVOT tachycardia, at least in its initiation. (+info)Ventricular Premature Complexes (VPCs), also known as Ventricular Extrasystoles or Premature Ventricular Contractions (PVCs), are extra heartbeats that originate in the ventricles, the lower chambers of the heart. These premature beats disrupt the normal sequence of electrical impulses in the heart and cause the ventricles to contract earlier than they should.
VPCs can result in a noticeable "skipped" or "extra" beat sensation, often followed by a stronger beat as the heart returns to its regular rhythm. They may occur occasionally in healthy individuals with no underlying heart condition, but frequent VPCs could indicate an underlying issue such as heart disease, electrolyte imbalance, or digitalis toxicity. In some cases, VPCs can be harmless and require no treatment; however, if they are frequent or associated with structural heart problems, further evaluation and management may be necessary to prevent potential complications like reduced cardiac output or heart failure.
Atrial premature complexes (APCs or APCTs) are extra heartbeats that originate in the atria, which are the upper chambers of the heart. These early beats disrupt the normal rhythm and cause a premature contraction before the next scheduled beat. APCs can sometimes be felt as a "skipped" beat or palpitation. They are usually benign and do not require treatment unless they occur frequently or are associated with underlying heart disease.
Moricizine is an antiarrhythmic medication that belongs to the class IC. It works by stabilizing the heart's electrical activity and correcting irregular heart rhythms (arrhythmias). Moricizine is used to treat certain types of serious, life-threatening ventricular arrhythmias.
It's important to note that moricizine has been discontinued in many countries due to the availability of safer and more effective antiarrhythmic medications. The use of moricizine should be under the close supervision of a healthcare professional, and it is usually reserved for situations where other treatments have not been effective.
The medical definition of 'Moricizine' is:
A class IC antiarrhythmic drug that prolongs the refractory period of the ventricles by selectively blocking sodium channels during phase 0 of the action potential, thereby stabilizing cardiac membranes and suppressing ectopic pacemaker activity. Moricizine is used in the treatment of certain types of serious, life-threatening ventricular arrhythmias.
Ambulatory electrocardiography, also known as ambulatory ECG or Holter monitoring, is a non-invasive method of recording the electrical activity of the heart over an extended period of time (typically 24 hours or more) while the patient goes about their daily activities. The device used to record the ECG is called a Holter monitor, which consists of a small, portable recorder that is attached to the patient's chest with electrodes.
The recorded data provides information on any abnormalities in the heart's rhythm or electrical activity during different stages of activity and rest, allowing healthcare providers to diagnose and evaluate various cardiac conditions such as arrhythmias, ischemia, and infarction. The ability to monitor the heart's activity over an extended period while the patient performs their normal activities provides valuable information that may not be captured during a standard ECG, which only records the heart's electrical activity for a few seconds.
In summary, ambulatory electrocardiography is a diagnostic tool used to evaluate the electrical activity of the heart over an extended period, allowing healthcare providers to diagnose and manage various cardiac conditions.
Cardiac arrhythmias are abnormal heart rhythms that result from disturbances in the electrical conduction system of the heart. The heart's normal rhythm is controlled by an electrical signal that originates in the sinoatrial (SA) node, located in the right atrium. This signal travels through the atrioventricular (AV) node and into the ventricles, causing them to contract and pump blood throughout the body.
An arrhythmia occurs when there is a disruption in this electrical pathway or when the heart's natural pacemaker produces an abnormal rhythm. This can cause the heart to beat too fast (tachycardia), too slow (bradycardia), or irregularly.
There are several types of cardiac arrhythmias, including:
1. Atrial fibrillation: A rapid and irregular heartbeat that starts in the atria (the upper chambers of the heart).
2. Atrial flutter: A rapid but regular heartbeat that starts in the atria.
3. Supraventricular tachycardia (SVT): A rapid heartbeat that starts above the ventricles, usually in the atria or AV node.
4. Ventricular tachycardia: A rapid and potentially life-threatening heart rhythm that originates in the ventricles.
5. Ventricular fibrillation: A chaotic and disorganized electrical activity in the ventricles, which can be fatal if not treated immediately.
6. Heart block: A delay or interruption in the conduction of electrical signals from the atria to the ventricles.
Cardiac arrhythmias can cause various symptoms, such as palpitations, dizziness, shortness of breath, chest pain, and fatigue. In some cases, they may not cause any symptoms and go unnoticed. However, if left untreated, certain types of arrhythmias can lead to serious complications, including stroke, heart failure, or even sudden cardiac death.
Treatment for cardiac arrhythmias depends on the type, severity, and underlying causes. Options may include lifestyle changes, medications, cardioversion (electrical shock therapy), catheter ablation, implantable devices such as pacemakers or defibrillators, and surgery. It is essential to consult a healthcare professional for proper evaluation and management of cardiac arrhythmias.
Electrocardiography (ECG or EKG) is a medical procedure that records the electrical activity of the heart. It provides a graphic representation of the electrical changes that occur during each heartbeat. The resulting tracing, called an electrocardiogram, can reveal information about the heart's rate and rhythm, as well as any damage to its cells or abnormalities in its conduction system.
During an ECG, small electrodes are placed on the skin of the chest, arms, and legs. These electrodes detect the electrical signals produced by the heart and transmit them to a machine that amplifies and records them. The procedure is non-invasive, painless, and quick, usually taking only a few minutes.
ECGs are commonly used to diagnose and monitor various heart conditions, including arrhythmias, coronary artery disease, heart attacks, and electrolyte imbalances. They can also be used to evaluate the effectiveness of certain medications or treatments.
Purkinje fibers are specialized cardiac muscle fibers that are located in the subendocardial region of the inner ventricular walls of the heart. They play a crucial role in the electrical conduction system of the heart, transmitting electrical impulses from the bundle branches to the ventricular myocardium, which enables the coordinated contraction of the ventricles during each heartbeat.
These fibers have a unique structure that allows for rapid and efficient conduction of electrical signals. They are larger in diameter than regular cardiac muscle fibers, have fewer branching points, and possess more numerous mitochondria and a richer blood supply. These features enable Purkinje fibers to conduct electrical impulses at faster speeds, ensuring that the ventricles contract simultaneously and forcefully, promoting efficient pumping of blood throughout the body.
Heart failure is a pathophysiological state in which the heart is unable to pump sufficient blood to meet the metabolic demands of the body or do so only at the expense of elevated filling pressures. It can be caused by various cardiac disorders, including coronary artery disease, hypertension, valvular heart disease, cardiomyopathy, and arrhythmias. Symptoms may include shortness of breath, fatigue, and fluid retention. Heart failure is often classified based on the ejection fraction (EF), which is the percentage of blood that is pumped out of the left ventricle during each contraction. A reduced EF (less than 40%) is indicative of heart failure with reduced ejection fraction (HFrEF), while a preserved EF (greater than or equal to 50%) is indicative of heart failure with preserved ejection fraction (HFpEF). There is also a category of heart failure with mid-range ejection fraction (HFmrEF) for those with an EF between 40-49%.
An encyclopedia is a comprehensive reference work containing articles on various topics, usually arranged in alphabetical order. In the context of medicine, a medical encyclopedia is a collection of articles that provide information about a wide range of medical topics, including diseases and conditions, treatments, tests, procedures, and anatomy and physiology. Medical encyclopedias may be published in print or electronic formats and are often used as a starting point for researching medical topics. They can provide reliable and accurate information on medical subjects, making them useful resources for healthcare professionals, students, and patients alike. Some well-known examples of medical encyclopedias include the Merck Manual and the Stedman's Medical Dictionary.
Heart valve diseases are a group of conditions that affect the function of one or more of the heart's four valves (tricuspid, pulmonic, mitral, and aortic). These valves are responsible for controlling the direction and flow of blood through the heart. Heart valve diseases can cause the valves to become narrowed (stenosis), leaky (regurgitation or insufficiency), or improperly closed (prolapse), leading to disrupted blood flow within the heart and potentially causing symptoms such as shortness of breath, fatigue, chest pain, and irregular heart rhythms. The causes of heart valve diseases can include congenital defects, age-related degenerative changes, infections, rheumatic heart disease, and high blood pressure. Treatment options may include medications, surgical repair or replacement of the affected valve(s), or transcatheter procedures.
Dilated cardiomyopathy (DCM) is a type of cardiomyopathy characterized by the enlargement and weakened contraction of the heart's main pumping chamber (the left ventricle). This enlargement and weakness can lead to symptoms such as shortness of breath, fatigue, and fluid retention. DCM can be caused by various factors including genetics, viral infections, alcohol and drug abuse, and other medical conditions like high blood pressure and diabetes. It is important to note that this condition can lead to heart failure if left untreated.
Heart valves are specialized structures in the heart that ensure unidirectional flow of blood through its chambers during the cardiac cycle. There are four heart valves: the tricuspid valve and the mitral (bicuspid) valve, located between the atria and ventricles, and the pulmonic (pulmonary) valve and aortic valve, located between the ventricles and the major blood vessels leaving the heart.
The heart valves are composed of thin flaps of tissue called leaflets or cusps, which are supported by a fibrous ring. The aortic and pulmonic valves have three cusps each, while the tricuspid and mitral valves have three and two cusps, respectively.
The heart valves open and close in response to pressure differences across them, allowing blood to flow forward into the ventricles during diastole (filling phase) and preventing backflow of blood into the atria during systole (contraction phase). A properly functioning heart valve ensures efficient pumping of blood by the heart and maintains normal blood circulation throughout the body.
Hypertrophic cardiomyopathy (HCM) is a genetic disorder characterized by the thickening of the heart muscle, specifically the ventricles (the lower chambers of the heart that pump blood out to the body). This thickening can make it harder for the heart to pump blood effectively, which can lead to symptoms such as shortness of breath, chest pain, and fatigue. In some cases, HCM can also cause abnormal heart rhythms (arrhythmias) and may increase the risk of sudden cardiac death.
The thickening of the heart muscle in HCM is caused by an overgrowth of the cells that make up the heart muscle, known as cardiomyocytes. This overgrowth can be caused by mutations in any one of several genes that encode proteins involved in the structure and function of the heart muscle. These genetic mutations are usually inherited from a parent, but they can also occur spontaneously in an individual with no family history of the disorder.
HCM is typically diagnosed using echocardiography (a type of ultrasound that uses sound waves to create images of the heart) and other diagnostic tests such as electrocardiogram (ECG) and cardiac magnetic resonance imaging (MRI). Treatment for HCM may include medications to help manage symptoms, lifestyle modifications, and in some cases, surgical procedures or implantable devices to help prevent or treat arrhythmias.
Blood pressure is the force exerted by circulating blood on the walls of the blood vessels. It is measured in millimeters of mercury (mmHg) and is given as two figures:
1. Systolic pressure: This is the pressure when the heart pushes blood out into the arteries.
2. Diastolic pressure: This is the pressure when the heart rests between beats, allowing it to fill with blood.
Normal blood pressure for adults is typically around 120/80 mmHg, although this can vary slightly depending on age, sex, and other factors. High blood pressure (hypertension) is generally considered to be a reading of 130/80 mmHg or higher, while low blood pressure (hypotension) is usually defined as a reading below 90/60 mmHg. It's important to note that blood pressure can fluctuate throughout the day and may be affected by factors such as stress, physical activity, and medication use.
Premature ventricular contraction
Dilated cardiomyopathy
Ashman phenomenon
List of dog diseases
Biofeedback
Lorcainide
Arrhythmogenic cardiomyopathy
Boxer cardiomyopathy
List of MeSH codes (C23)
Cardiac Arrhythmia Suppression Trial
List of MeSH codes (C14)
Premature junctional contraction
Sodium channel blocker
Ventricular escape beat
Bradycardia
Junctional escape beat
Atrioventricular reentrant tachycardia
Ectopic pacemaker
Electrical alternans
Lown-Ganong-Levine syndrome
Premature atrial contraction
Outline of cardiology
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Hein Wellens
Ventricular Premature Complexes: Background, Pathophysiology, Etiology
Mechanical suppression of premature ventricular complexes during catheter ablation procedures<...
Premature Ventricular Complex Archives - ECG Made Simple
Premature ventricular contraction - Wikipedia
Obesity and exercise-induced ectopic ventricular arrhythmias in apparently healthy middle aged adults
Characteristics of diurnal ventricular premature complex variation in right ventricular outflow tract arrhythmias after...
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Tachycardia10
- Observational studies and inferences from typical electrophysiology studies were initially focused on ventricular ectopy triggering ventricular tachycardia (VT), which, in turn, can degenerate into ventricular fibrillation, as a mechanisms for sudden cardiac death. (medscape.com)
- We investigated the association between body mass index (BMI) and the risk for ectopic ventricular activity (defined as multiple ventricular premature beats (≥3), ventricular bigeminy, nonsustained ventricular tachycardia or sustained ventricular tachycardia) during exercise stress testing among 22,516 apparently healthy men and women who attended periodic health screening examinations between the years 2000 and 2014. (nih.gov)
- Atrial premature complexes (APC), ventricular premature complexes (VPC), and supraventricular tachyarrhythmias are more prevalent than ventricular tachycardia, especially in women without structural heart disease. (hindawi.com)
- Following myocardial infarction (MI), the myocardium is prone to calcium-driven alternans, which typically precedes ventricular tachycardia and fibrillation. (frontiersin.org)
- Ventricular tachycardia is defined as three or more consecutive ventricular premature beats. (health.am)
- Ventricular tachycardia is either nonsustained (lasting less than 30 seconds) or sustained. (health.am)
- Ventricular tachycardia is a frequent complication of acute myocardial infarction and dilated cardiomyopathy but may occur in chronic coronary disease, hypertrophic cardiomyopathy, mitral valve prolapse, myocarditis, and in most other forms of myocardial disease. (health.am)
- In nonacute settings, most patients with ventricular tachycardia have known or easily detectable cardiac disease, and the finding of ventricular tachycardia is an unfavorable prognostic sign. (health.am)
- 90 ventricular premature complexes (VPCs) and/or nonsustained ventricular tachycardia (VT) on 24-hour Holter monitoring were randomly divided into 2 groups. (nih.gov)
- Can adjustments in voltage criteria unmask conducting channels in patients with post-infarction ventricular tachycardia? (escardio.org)
Fibrillation9
- Atrial fibrillation, flutter and ventricular premature complexes can also be found. (escardio.org)
- ECM is emerging as an important tool not only for diagnostic mapping, but also as a guide for trans-catheter ablation of complex arrhythmias such as atrial fibrillation. (minervamedica.it)
- Panoramic mapping of ventricular fibrillation (VF) is helping to develop a better understanding of its physiology, with future implications for the identification of therapeutic targets in patients with structural heart disease, as well as in idiopathic VF. (minervamedica.it)
- Sudden death occurs more frequently (presumably as a result of ventricular fibrillation ) when ventricular premature beats occur in the presence of organic heart disease but not in individuals with no known cardiac disease. (health.am)
- Of total 48 patients, the current study showed in the subgroup of 26 patients with OSA a more prevalent presence of ACDs including premature atrial complexes, sinus bradycardia, sinus pauses, premature ventricular complexes and paroxysmal atrial fibrillation,compared to otherwise similar subgroup of 22 patients without OSA. (easychair.org)
- Alpha receptor blockade with either phentolamine or prazosin significantly reduced the number of premature ventricular complexes during coronary reperfusion (321 +/- 62-14 +/- 10 premature ventricular complexes, P less than 0.001), abolished early ventricular fibrillation (from 25% in controls to 0%), and prevented the increase in idioventricular rate seen with coronary reperfusion. (jci.org)
- Electrical Substrate Ablation for Refractory Ventricular Fibrillation: Results of the AVATAR Study. (stanford.edu)
- Background - Refractory ventricular fibrillation (VF) is a challenging clinical entity, for which ablation of triggering premature ventricular complexes (PVCs) is described. (stanford.edu)
- CARL pioneered computational mapping of 'chaotic' heart rhythm disturbances (arrhythmias), targeting mechanistic drivers for atrial and ventricular fibrillation (AF, VF) using machine learning and computational models. (stanford.edu)
Arrhythmias8
- Multivariate binary logistic regression with repeated measures of 92,619 ESTs, showed that obese subjects were 33% more likely to have ectopic ventricular arrhythmias during exercise compared with subjects with normal weight (p = 0.005), and that each 1 kg/m(2) increase in BMI was associated with a significant 4% (p = 0.002) increased adjusted risk for exercise-induced ventricular arrhythmias. (nih.gov)
- Premature extra beats and sustained tachyarrhythmias are the most common arrhythmias in pregnancy. (hindawi.com)
- Disopyramide is a prescription medications used to treat life-threatening ventricular (lower chambers of the heart) arrhythmias. (rxwiki.com)
- Premature ventricular contractions (PVCs) are common arrhythmias, most of them requiring no follow-up or treatment. (dergisi.org)
- We concluded that serum TSH concentration has a role in the frequency of PVC, therefore, SH may lead to a predisposition to ventricular arrhythmias. (dergisi.org)
- The class I and III agents are all effective in reducing ventricular premature beats but often cause side effects and may exacerbate serious arrhythmias in 5-20% of patients. (health.am)
- Specific support has been via grants HL70529 and HL162260 for ventricular arrhythmias, and HL103800, HL83359, HL122384, HL149134, and several SBIR grants for atrial arrhythmias. (stanford.edu)
- vided that no complex arrhythmias exist. (lu.se)
PVCs10
- Introduction: Mechanical suppression of premature ventricular complexes (PVCs) is not a well-known observation. (psu.edu)
- Premature ventricular contractions (PVCs) are extra heartbeats that begin in one of the heart's two lower pumping chambers (ventricles). (nchmd.org)
- To understand the cause of premature ventricular contractions (PVCs), it might help to learn more about how the heart typically beats. (nchmd.org)
- Certain lifestyle choices and health conditions may make a person more likely to develop premature ventricular contractions (PVCs). (nchmd.org)
- Idiopathic premature ventricular complexes (PVCs) are considered a benign form of cardiac arrhythmia. (juniperpublishers.com)
- However, several reports demonstrated that frequent PVCs were associated with left ventricular (LV) dysfunction, increased LV dimensions and cardiomyopathy. (juniperpublishers.com)
- Electrophysiology study using the Carto® 3 System (Biosense Webster, Irvine, CA) showed PVCs arising from anteroseptal right ventricular outflow tract. (juniperpublishers.com)
- 3 ] in 1998 had shown that premature ventricular complexes (PVCs) was associated with cardiomyopathy and pharmacological suppression of PVCs in patients with presumed idiopathic dilated cardiomyopathy subsequently improved left ventricular (LV) systolic dysfunction. (juniperpublishers.com)
- She was diagnosed with dilated cardiomyopathy based on echocardiography that showed a reduced left ventricular ejection fraction (LVEF) of 25% and LV systolic diameter 5.2cm with mild functional mitral regurgitation (MR). Her baseline 12-lead ECG showed inferior axis PVCs with left bundle branch block morphology ( Figure 1 ). (juniperpublishers.com)
- Our article is an excellent reference point to learn a little more about premature ventricular complexes (PVCs) so that you can recognise the differences between this condition and others which are similar. (drsanjayguptacardiologist.com)
Arrhythmia7
- VPCs are the most common ventricular arrhythmia. (medscape.com)
- In animal models, focal mechanisms without evidence of macro-reentry play a major role in the origin of ventricular arrhythmia associated with ischemic cardiomyopathy. (medscape.com)
- We retrospectively reviewed this phenomenon in the Ventricular Arrhythmia (VA) ablation procedures performed at Richard L. Roudebush Veterans Health Administration (VHA) center. (psu.edu)
- Obesity is independently associated with increased likelihood of ectopic ventricular arrhythmia during exercise. (nih.gov)
- LMNA and PLN mutation carriers showed a high prevalence of cardiac transplantation and ventricular arrhythmia. (wikipedia.org)
- Premature ventricular contractions are a common type of irregular heartbeat (arrhythmia). (nchmd.org)
- We searched on Pubmed for ventricular premature complexes (VPC) in pregnant women and found no case reporting increased incidence of this arrhythmia while supine. (hindawi.com)
Contractions6
- Autoimmune mechanisms Thiamine deficiency Recent studies have shown that those subjects with an extremely high occurrence (several thousands a day) of premature ventricular contractions (extrasystole) can develop dilated cardiomyopathy. (wikipedia.org)
- The diurnal variation type (group 1, n = 36) was defined as those patients who had most ventricular premature contractions (VPCs) during the night hours by preprocedure Holter recordings. (tmu.edu.tw)
- Occasional premature ventricular contractions in people without heart disease usually aren't a concern and likely don't need treatment. (nchmd.org)
- You might need treatment if the premature ventricular contractions are very frequent or bothersome, or if you have an underlying heart condition. (nchmd.org)
- Premature ventricular contractions often cause few or no symptoms. (nchmd.org)
- The cause of premature ventricular contractions isn't always clear. (nchmd.org)
Catheter1
- Activation mapping with a contact-force ablation catheter (7.5 French, ThermoCool® SmartTouch® Catheter, Biosense-Webster) was performed and the earliest activation was detected from the anteroseptal right ventricular outflow tract ( Figure 2 ). (juniperpublishers.com)
Right ventricular outfl1
- The electrophysiological characteristics of diurnal variations in idiopathic right ventricular outflow tract (RVOT) VA have not previously been elucidated. (tmu.edu.tw)
Cardiac2
- Mitral regurgitation is related to posterior papillary muscle dysfunction.Additionally, the sudden onset of cardiac symptoms accompanied by deterioration of left ventricular function in young DMD patients already on treatment with a prednisone derivate, that postpones the development of DMD cardiomyopathy, should raise the suspicion of possible myocarditis (3). (escardio.org)
- The purpose of this clinical study is to test the hypothesis that market released Cardiac Resynchronization Therapy (CRT) devices which contain the AdaptivCRT® (aCRT) algorithm have a superior outcome compared to standard CRT devices in CRT indicated patients with normal atrio-ventricular (AV) conduction and left bundle branch block (LBBB). (stanford.edu)
Sinus bradycardia1
- Electrocardiogram (ECG) results demonstrated sinus bradycardia, frequent premature ventricular complexes, and scooped ST segments consistent with cardenolide toxicity. (cdc.gov)
Cardiomyopathy3
- VPCs also are common in patients with hypertension, ventricular hypertrophy, cardiomyopathy, and mitral valve prolapse. (medscape.com)
- If the underlying condition is mitral prolapse, hypertrophic cardiomyopathy, left ventricular hypertrophy, or coronary disease - or if the QT interval is prolonged - ß-blocker therapy is appropriate. (health.am)
- Nandakumar R, Koh K W, Alan K C C, Yap L B, Razali O, Zulkeflee M. Successful Treatment of Cardiomyopathy Induced by Premature Ventricular Complexes. (juniperpublishers.com)
Idiopathic1
- Diurnal variations in ventricular tachyarrhythmias (VAs) have been demonstrated in idiopathic arrhythmogenic heart disease. (tmu.edu.tw)
Atrial and ventricular1
- Treatment options range from medication to atrial and ventricular ablation to pacemaker and defibrillator implantation (ICD) and the WATCHMAN ™ device. (uchealth.org)
Tachyarrhythmias1
- Some patients are aware of virtually every premature ventricular beat, but others are unaware of even complex atrial or ventricular tachyarrhythmias. (msdmanuals.com)
Ectopy1
- Frolkis JP et al: Frequent ventricular ectopy after exercise as a predictor of death. (health.am)
Frequent2
- Ambulatory electrocardiographic monitoring or monitoring during graded exercise may reveal more frequent and complex ventricular premature beats than occur in a single routine ECG. (health.am)
- It was previously thought that frequent ventricular complexes in patients without structural heart disease was considered to be a benign condition without prognostic significance [ 1 , 2 ]. (juniperpublishers.com)
Ectopic ventricular1
- However, there are limited data on the association between excess weight and the risk of ectopic ventricular activity. (nih.gov)
Pacemaker2
- nbsp;When the conduction impulses from SA nodes do not reach the ventricles for some reason or when the rate of these impulses falls below the base rate determined by the ventricular pacemaker cells, the impulses are generated by lower heart region, resulting in idioventricular rhythm or ventricle escape complexes commonly known as irregular heartbeats. (petyourdog.com)
- A decrese in the frequency of sinus node pacemaker impulses or its blockage to the ventricles results in taking over of pacemaker role by lower heart region, which results in ventricular escape complexes or an idioventricular rhythm. (petyourdog.com)
Conduction2
- Simulations also demonstrated that β-adrenergic receptor stimulation in this specific region reduced the formation of conduction block and the probability of premature ventricular activation propagation. (frontiersin.org)
- Les troubles du rythme et de la conduction cardiaque peuvent mettre en jeu le pronostic vital à court terme au cours de la COVID -19. (bvsalud.org)
Extrasystoles1
- In cases of isolated extrasystoles, the patient may actually perceive the augmented postextrasystolic beat as the "skipped" beat rather than the premature beat itself, probably because the extrasystole blocks the next sinus beat and allows longer ventricular filling and thus a higher stroke volume. (msdmanuals.com)
VPCs3
- Ventricular premature complexes (VPCs) are ectopic impulses originating from an area distal to the His Purkinje system. (medscape.com)
- Assessment and treatment of VPCs is challenging and complex, and is highly dependent on the clinical context. (medscape.com)
- Symptoms, a 6-minute walk test, left ventricular (LV) ejection fraction (EF), frequency and complexity of VPCs, and quality of life were assessed after 8 weeks of treatment and during a mean 24-month follow-up. (nih.gov)
Coronary1
- Ventricular dysrhythmias induced by coronary occlusion alone (without reperfusion) were attenuated markedly by alpha-receptor blockade under conditions in which perfusion (measured with radiolabeled microspheres) within ischemic zones was not affected. (jci.org)
Exercise-induced1
- Jouven X et al: Long-term outcome in asymptomatic men with exercise-induced premature ventricular depolarizations. (health.am)
Depolarization3
- Automaticity: This is the development of a new site of depolarization in nonnodal ventricular tissue, which can lead to a VPC. (medscape.com)
- The P wave represents atrial depolarization and the QRS represents ventricular depolarization. (petyourdog.com)
- Notice the unconducted atrial premature complexes (APCs) superimposed on the ST segment of the previous depolarization (arrows) that appear in a bigeminal pattern. (avma.org)
Beats3
- Ventricular premature beats are characterized by wide QRS complexes that differ in morphology from the patient's normal beats. (health.am)
- Exercise generally abolishes premature beats in normal hearts, and the rhythm becomes regular. (health.am)
- An increased frequency of ventricular premature beats during exercise is associated with a higher risk of cardiovascular mortality, though there is no evidence that specific therapy has a role. (health.am)
Holter1
- Heart rate (a) and ventricular premature complexes (b) during 24-hour Holter monitoring. (hindawi.com)
Left1
- We also consult with the Advanced Heart Failure team at UCHealth University of Colorado Hospital when we have patients who might need LVAD (left ventricular assist device) or a heart transplant. (uchealth.org)
Rhythm1
- In dogs with idioventricular rhythm, the P wave is either absent or hidden behind the QRS complex. (petyourdog.com)
Heartbeat1
- When your heartbeat does't follow the typical heartbeat pattern and instead is slightly offbeat, it's called a premature ventricular complex, or PVC. (healthline.com)