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Ventricular Function, LeftVentricular Function, RightVentricular Dysfunction, LeftVentricular FunctionStroke VolumeHeart VentriclesEchocardiographyHeartMyocardial ContractionMyocardial InfarctionVentricular Dysfunction, RightSystoleHemodynamicsDiastoleRadionuclide VentriculographyHeart FailureMyocardiumGated Blood-Pool ImagingElectrocardiographyRadionuclide AngiographyEchocardiography, DopplerCoronary CirculationHeart Function TestsHypertrophy, Left VentricularCardiomyopathy, DilatedCardiac CatheterizationVentricular RemodelingCineangiographyTime FactorsCoronary DiseaseHeart RateCardiac VolumeVentriculography, First-PassVentricular PressureCardiac OutputCardiomyopathiesEchocardiography, Doppler, PulsedMyocardial IschemiaTreatment OutcomeDobutamineFollow-Up StudiesAngiocardiographyExercise TestBlood PressureAtrial Function, LeftProspective StudiesCardiotonic AgentsVentricular DysfunctionCoronary AngiographyMagnetic Resonance Imaging, CineCoronary Artery BypassEchocardiography, Doppler, ColorCardiac Pacing, ArtificialTomography, Emission-Computed, Single-PhotonMitral Valve InsufficiencyPredictive Value of TestsPhonocardiographyMyocardial ReperfusionDogsMitral ValveGold RadioisotopesHeart DiseasesCoronary VesselsAortic Valve StenosisTricuspid ValveMyocardial Reperfusion InjuryAngioplasty, Balloon, CoronaryTechnetium Tc 99m SestamibiAngina PectorisAdrenergic beta-AntagonistsEchocardiography, StressPulmonary ArteryNatriuretic Peptide, BrainPrognosisRecovery of FunctionKinetocardiographyTetralogy of FallotCardiac Surgical ProceduresCineradiographyHeart AtriaTransposition of Great VesselsMyocardial StunningHeart AneurysmVentricular SeptumStreptokinaseBlood Flow VelocityArrhythmias, CardiacDisease Models, AnimalMyocardial RevascularizationTissue SurvivalPacemaker, ArtificialHeart SeptumHeart Valve Prosthesis ImplantationObserver VariationMetoprololEndocardiumCardiac Output, LowExercise ToleranceHeart Valve ProsthesisMyocarditisAnalysis of VarianceHeart Defects, CongenitalMyocytes, CardiacAortic ValveHypertrophy, Right VentricularChronic DiseaseAtrial FibrillationReproducibility of ResultsCoronary Artery DiseasePulmonary Valve InsufficiencySeverity of Illness IndexHypertension, PulmonaryThrombolytic TherapyElectrocardiography, AmbulatoryPostoperative PeriodRadiopharmaceuticalsPhysical ExertionPropanolaminesVascular ResistanceHeart Arrest, InducedCardiomegalyRetrospective StudiesThallium RadioisotopesHeart-Assist DevicesCardiomyopathy, HypertrophicAortic Valve InsufficiencyMagnetic Resonance ImagingOxygen ConsumptionHypoplastic Left Heart SyndromeFibrosisPostoperative ComplicationsCollateral CirculationTrimetazidineEchocardiography, TransesophagealPulmonary Wedge PressureCardiovascular AgentsEdema, CardiacTachycardia, VentricularFeasibility StudiesEchocardiography, Three-DimensionalAngiotensin-Converting Enzyme InhibitorsContrast MediaPericardiumFontan ProcedureRisk FactorsOrgan SizeRestHeart Valve DiseasesAnterior Wall Myocardial InfarctionCardiopulmonary BypassHeart BlockSurvival RateRandom AllocationSensitivity and SpecificityLinear ModelsCardiovascular Surgical ProceduresCoronary Vessel AnomaliesMyocardial Perfusion ImagingCarbazolesHeart TransplantationIodobenzenesImage Processing, Computer-AssistedTachycardiaHeart SoundsTomography, Emission-ComputedBundle-Branch BlockDouble-Blind MethodCase-Control StudiesTantalumOrganotechnetium CompoundsHypertensionDefibrillators, ImplantableBiological MarkersTomography, X-Ray ComputedPericardial EffusionCardioplegic SolutionsRats, WistarPressureAdrenergic beta-AgonistsMitral Valve StenosisAtrial Natriuretic FactorTricuspid Valve InsufficiencyEndocardial FibroelastosisAdrenergic beta-1 Receptor AgonistsRats, Sprague-DawleyPerfusionTakotsubo CardiomyopathyAnti-Arrhythmia AgentsVasodilator AgentsAcute DiseaseRisk AssessmentSwineIntra-Aortic Balloon PumpingCoronary StenosisStatistics, NonparametricModels, CardiovascularExtracorporeal CirculationScintillation CountingChi-Square DistributionAngiographyTechnetium
Ventricular Function, Left: The hemodynamic and electrophysiological action of the left HEART VENTRICLE. Its measurement is an important aspect of the clinical evaluation of patients with heart disease to determine the effects of the disease on cardiac performance.Ventricular Function, Right: The hemodynamic and electrophysiological action of the right HEART VENTRICLE.Ventricular Dysfunction, Left: A condition in which the LEFT VENTRICLE of the heart was functionally impaired. This condition usually leads to HEART FAILURE; MYOCARDIAL INFARCTION; and other cardiovascular complications. Diagnosis is made by measuring the diminished ejection fraction and a depressed level of motility of the left ventricular wall.Ventricular Function: The hemodynamic and electrophysiological action of the HEART VENTRICLES.Stroke Volume: The amount of BLOOD pumped out of the HEART per beat, not to be confused with cardiac output (volume/time). It is calculated as the difference between the end-diastolic volume and the end-systolic volume.Heart Ventricles: The lower right and left chambers of the heart. The right ventricle pumps venous BLOOD into the LUNGS and the left ventricle pumps oxygenated blood into the systemic arterial circulation.Echocardiography: Ultrasonic recording of the size, motion, and composition of the heart and surrounding tissues. The standard approach is transthoracic.Heart: The hollow, muscular organ that maintains the circulation of the blood.Myocardial Contraction: Contractile activity of the MYOCARDIUM.Myocardial Infarction: NECROSIS of the MYOCARDIUM caused by an obstruction of the blood supply to the heart (CORONARY CIRCULATION).Ventricular Dysfunction, Right: A condition in which the RIGHT VENTRICLE of the heart was functionally impaired. This condition usually leads to HEART FAILURE or MYOCARDIAL INFARCTION, and other cardiovascular complications. Diagnosis is made by measuring the diminished ejection fraction and a depressed level of motility of the right ventricular wall.Systole: Period of contraction of the HEART, especially of the HEART VENTRICLES.Hemodynamics: The movement and the forces involved in the movement of the blood through the CARDIOVASCULAR SYSTEM.Diastole: Post-systolic relaxation of the HEART, especially the HEART VENTRICLES.Radionuclide Ventriculography: Imaging of a ventricle of the heart after the injection of a radioactive contrast medium. The technique is less invasive than cardiac catheterization and is used to assess ventricular function.Heart Failure: A heterogeneous condition in which the heart is unable to pump out sufficient blood to meet the metabolic need of the body. Heart failure can be caused by structural defects, functional abnormalities (VENTRICULAR DYSFUNCTION), or a sudden overload beyond its capacity. Chronic heart failure is more common than acute heart failure which results from sudden insult to cardiac function, such as MYOCARDIAL INFARCTION.Myocardium: The muscle tissue of the HEART. It is composed of striated, involuntary muscle cells (MYOCYTES, CARDIAC) connected to form the contractile pump to generate blood flow.Gated Blood-Pool Imaging: Radionuclide ventriculography where scintigraphic data is acquired during repeated cardiac cycles at specific times in the cycle, using an electrocardiographic synchronizer or gating device. Analysis of right ventricular function is difficult with this technique; that is best evaluated by first-pass ventriculography (VENTRICULOGRAPHY, FIRST-PASS).Electrocardiography: Recording of the moment-to-moment electromotive forces of the HEART as projected onto various sites on the body's surface, delineated as a scalar function of time. The recording is monitored by a tracing on slow moving chart paper or by observing it on a cardioscope, which is a CATHODE RAY TUBE DISPLAY.Radionuclide Angiography: The measurement of visualization by radiation of any organ after a radionuclide has been injected into its blood supply. It is used to diagnose heart, liver, lung, and other diseases and to measure the function of those organs, except renography, for which RADIOISOTOPE RENOGRAPHY is available.Echocardiography, Doppler: Measurement of intracardiac blood flow using an M-mode and/or two-dimensional (2-D) echocardiogram while simultaneously recording the spectrum of the audible Doppler signal (e.g., velocity, direction, amplitude, intensity, timing) reflected from the moving column of red blood cells.Coronary Circulation: The circulation of blood through the CORONARY VESSELS of the HEART.Heart Function Tests: Examinations used to diagnose and treat heart conditions.Hypertrophy, Left Ventricular: Enlargement of the LEFT VENTRICLE of the heart. This increase in ventricular mass is attributed to sustained abnormal pressure or volume loads and is a contributor to cardiovascular morbidity and mortality.Cardiomyopathy, Dilated: A form of CARDIAC MUSCLE disease that is characterized by ventricular dilation, VENTRICULAR DYSFUNCTION, and HEART FAILURE. Risk factors include SMOKING; ALCOHOL DRINKING; HYPERTENSION; INFECTION; PREGNANCY; and mutations in the LMNA gene encoding LAMIN TYPE A, a NUCLEAR LAMINA protein.Cardiac Catheterization: Procedures in which placement of CARDIAC CATHETERS is performed for therapeutic or diagnostic procedures.Ventricular Remodeling: The geometric and structural changes that the HEART VENTRICLES undergo, usually following MYOCARDIAL INFARCTION. It comprises expansion of the infarct and dilatation of the healthy ventricle segments. While most prevalent in the left ventricle, it can also occur in the right ventricle.Cineangiography: Motion pictures of the passage of contrast medium through blood vessels.Time Factors: Elements of limited time intervals, contributing to particular results or situations.Coronary Disease: An imbalance between myocardial functional requirements and the capacity of the CORONARY VESSELS to supply sufficient blood flow. It is a form of MYOCARDIAL ISCHEMIA (insufficient blood supply to the heart muscle) caused by a decreased capacity of the coronary vessels.Heart Rate: The number of times the HEART VENTRICLES contract per unit of time, usually per minute.Cardiac Volume: The volume of the HEART, usually relating to the volume of BLOOD contained within it at various periods of the cardiac cycle. The amount of blood ejected from a ventricle at each beat is STROKE VOLUME.Ventriculography, First-Pass: Radionuclide ventriculography where a bolus of radionuclide is injected and data are recorded from one pass through the heart ventricle. Left and right ventricular function can be analyzed independently during this technique. First-pass ventriculography is preferred over GATED BLOOD-POOL IMAGING for assessing right ventricular function.Ventricular Pressure: The pressure within a CARDIAC VENTRICLE. Ventricular pressure waveforms can be measured in the beating heart by catheterization or estimated using imaging techniques (e.g., DOPPLER ECHOCARDIOGRAPHY). The information is useful in evaluating the function of the MYOCARDIUM; CARDIAC VALVES; and PERICARDIUM, particularly with simultaneous measurement of other (e.g., aortic or atrial) pressures.Cardiac Output: The volume of BLOOD passing through the HEART per unit of time. It is usually expressed as liters (volume) per minute so as not to be confused with STROKE VOLUME (volume per beat).Cardiomyopathies: A group of diseases in which the dominant feature is the involvement of the CARDIAC MUSCLE itself. Cardiomyopathies are classified according to their predominant pathophysiological features (DILATED CARDIOMYOPATHY; HYPERTROPHIC CARDIOMYOPATHY; RESTRICTIVE CARDIOMYOPATHY) or their etiological/pathological factors (CARDIOMYOPATHY, ALCOHOLIC; ENDOCARDIAL FIBROELASTOSIS).Echocardiography, Doppler, Pulsed: Echocardiography applying the Doppler effect, with velocity detection combined with range discrimination. Short bursts of ultrasound are transmitted at regular intervals and the echoes are demodulated as they return.Myocardial Ischemia: A disorder of cardiac function caused by insufficient blood flow to the muscle tissue of the heart. The decreased blood flow may be due to narrowing of the coronary arteries (CORONARY ARTERY DISEASE), to obstruction by a thrombus (CORONARY THROMBOSIS), or less commonly, to diffuse narrowing of arterioles and other small vessels within the heart. Severe interruption of the blood supply to the myocardial tissue may result in necrosis of cardiac muscle (MYOCARDIAL INFARCTION).Treatment Outcome: Evaluation undertaken to assess the results or consequences of management and procedures used in combating disease in order to determine the efficacy, effectiveness, safety, and practicability of these interventions in individual cases or series.Dobutamine: A catecholamine derivative with specificity for BETA-1 ADRENERGIC RECEPTORS. It is commonly used as a cardiotonic agent after CARDIAC SURGERY and during DOBUTAMINE STRESS ECHOCARDIOGRAPHY.Follow-Up Studies: Studies in which individuals or populations are followed to assess the outcome of exposures, procedures, or effects of a characteristic, e.g., occurrence of disease.Angiocardiography: Radiography of the heart and great vessels after injection of a contrast medium.Exercise Test: Controlled physical activity which is performed in order to allow assessment of physiological functions, particularly cardiovascular and pulmonary, but also aerobic capacity. Maximal (most intense) exercise is usually required but submaximal exercise is also used.Blood Pressure: PRESSURE of the BLOOD on the ARTERIES and other BLOOD VESSELS.Atrial Function, Left: The hemodynamic and electrophysiological action of the LEFT ATRIUM.Prospective Studies: Observation of a population for a sufficient number of persons over a sufficient number of years to generate incidence or mortality rates subsequent to the selection of the study group.Cardiotonic Agents: Agents that have a strengthening effect on the heart or that can increase cardiac output. They may be CARDIAC GLYCOSIDES; SYMPATHOMIMETICS; or other drugs. They are used after MYOCARDIAL INFARCT; CARDIAC SURGICAL PROCEDURES; in SHOCK; or in congestive heart failure (HEART FAILURE).Ventricular Dysfunction: A condition in which HEART VENTRICLES exhibit impaired function.Coronary Angiography: Radiography of the vascular system of the heart muscle after injection of a contrast medium.Magnetic Resonance Imaging, Cine: A type of imaging technique used primarily in the field of cardiology. By coordinating the fast gradient-echo MRI sequence with retrospective ECG-gating, numerous short time frames evenly spaced in the cardiac cycle are produced. These images are laced together in a cinematic display so that wall motion of the ventricles, valve motion, and blood flow patterns in the heart and great vessels can be visualized.Coronary Artery Bypass: Surgical therapy of ischemic coronary artery disease achieved by grafting a section of saphenous vein, internal mammary artery, or other substitute between the aorta and the obstructed coronary artery distal to the obstructive lesion.Echocardiography, Doppler, Color: Echocardiography applying the Doppler effect, with the superposition of flow information as colors on a gray scale in a real-time image.Cardiac Pacing, Artificial: Regulation of the rate of contraction of the heart muscles by an artificial pacemaker.Tomography, Emission-Computed, Single-Photon: A method of computed tomography that uses radionuclides which emit a single photon of a given energy. The camera is rotated 180 or 360 degrees around the patient to capture images at multiple positions along the arc. The computer is then used to reconstruct the transaxial, sagittal, and coronal images from the 3-dimensional distribution of radionuclides in the organ. The advantages of SPECT are that it can be used to observe biochemical and physiological processes as well as size and volume of the organ. The disadvantage is that, unlike positron-emission tomography where the positron-electron annihilation results in the emission of 2 photons at 180 degrees from each other, SPECT requires physical collimation to line up the photons, which results in the loss of many available photons and hence degrades the image.Mitral Valve Insufficiency: Backflow of blood from the LEFT VENTRICLE into the LEFT ATRIUM due to imperfect closure of the MITRAL VALVE. This can lead to mitral valve regurgitation.Predictive Value of Tests: In screening and diagnostic tests, the probability that a person with a positive test is a true positive (i.e., has the disease), is referred to as the predictive value of a positive test; whereas, the predictive value of a negative test is the probability that the person with a negative test does not have the disease. Predictive value is related to the sensitivity and specificity of the test.Phonocardiography: Graphic registration of the heart sounds picked up as vibrations and transformed by a piezoelectric crystal microphone into a varying electrical output according to the stresses imposed by the sound waves. The electrical output is amplified by a stethograph amplifier and recorded by a device incorporated into the electrocardiograph or by a multichannel recording machine.Myocardial Reperfusion: Generally, restoration of blood supply to heart tissue which is ischemic due to decrease in normal blood supply. The decrease may result from any source including atherosclerotic obstruction, narrowing of the artery, or surgical clamping. Reperfusion can be induced to treat ischemia. Methods include chemical dissolution of an occluding thrombus, administration of vasodilator drugs, angioplasty, catheterization, and artery bypass graft surgery. However, it is thought that reperfusion can itself further damage the ischemic tissue, causing MYOCARDIAL REPERFUSION INJURY.Dogs: The domestic dog, Canis familiaris, comprising about 400 breeds, of the carnivore family CANIDAE. They are worldwide in distribution and live in association with people. (Walker's Mammals of the World, 5th ed, p1065)Mitral Valve: The valve between the left atrium and left ventricle of the heart.Gold Radioisotopes: Unstable isotopes of gold that decay or disintegrate emitting radiation. Au 185-196, 198-201, and 203 are radioactive gold isotopes.Heart Diseases: Pathological conditions involving the HEART including its structural and functional abnormalities.Coronary Vessels: The veins and arteries of the HEART.Aortic Valve Stenosis: A pathological constriction that can occur above (supravalvular stenosis), below (subvalvular stenosis), or at the AORTIC VALVE. It is characterized by restricted outflow from the LEFT VENTRICLE into the AORTA.Tricuspid Valve: The valve consisting of three cusps situated between the right atrium and right ventricle of the heart.Myocardial Reperfusion Injury: Damage to the MYOCARDIUM resulting from MYOCARDIAL REPERFUSION (restoration of blood flow to ischemic areas of the HEART.) Reperfusion takes place when there is spontaneous thrombolysis, THROMBOLYTIC THERAPY, collateral flow from other coronary vascular beds, or reversal of vasospasm.Angioplasty, Balloon, Coronary: Dilation of an occluded coronary artery (or arteries) by means of a balloon catheter to restore myocardial blood supply.Technetium Tc 99m Sestamibi: A technetium imaging agent used to reveal blood-starved cardiac tissue during a heart attack.Angina Pectoris: The symptom of paroxysmal pain consequent to MYOCARDIAL ISCHEMIA usually of distinctive character, location and radiation. It is thought to be provoked by a transient stressful situation during which the oxygen requirements of the MYOCARDIUM exceed that supplied by the CORONARY CIRCULATION.Adrenergic beta-Antagonists: Drugs that bind to but do not activate beta-adrenergic receptors thereby blocking the actions of beta-adrenergic agonists. Adrenergic beta-antagonists are used for treatment of hypertension, cardiac arrhythmias, angina pectoris, glaucoma, migraine headaches, and anxiety.Echocardiography, Stress: A method of recording heart motion and internal structures by combining ultrasonic imaging with exercise testing (EXERCISE TEST) or pharmacologic stress.Pulmonary Artery: The short wide vessel arising from the conus arteriosus of the right ventricle and conveying unaerated blood to the lungs.Natriuretic Peptide, Brain: A PEPTIDE that is secreted by the BRAIN and the HEART ATRIA, stored mainly in cardiac ventricular MYOCARDIUM. It can cause NATRIURESIS; DIURESIS; VASODILATION; and inhibits secretion of RENIN and ALDOSTERONE. It improves heart function. It contains 32 AMINO ACIDS.Prognosis: A prediction of the probable outcome of a disease based on a individual's condition and the usual course of the disease as seen in similar situations.Recovery of Function: A partial or complete return to the normal or proper physiologic activity of an organ or part following disease or trauma.Kinetocardiography: The graphic recording of chest wall movement due to cardiac impulses.Tetralogy of Fallot: A combination of congenital heart defects consisting of four key features including VENTRICULAR SEPTAL DEFECTS; PULMONARY STENOSIS; RIGHT VENTRICULAR HYPERTROPHY; and a dextro-positioned AORTA. In this condition, blood from both ventricles (oxygen-rich and oxygen-poor) is pumped into the body often causing CYANOSIS.Cardiac Surgical Procedures: Surgery performed on the heart.Cineradiography: Motion picture study of successive images appearing on a fluoroscopic screen.Heart Atria: The chambers of the heart, to which the BLOOD returns from the circulation.Transposition of Great Vessels: A congenital cardiovascular malformation in which the AORTA arises entirely from the RIGHT VENTRICLE, and the PULMONARY ARTERY arises from the LEFT VENTRICLE. Consequently, the pulmonary and the systemic circulations are parallel and not sequential, so that the venous return from the peripheral circulation is re-circulated by the right ventricle via aorta to the systemic circulation without being oxygenated in the lungs. This is a potentially lethal form of heart disease in newborns and infants.Myocardial Stunning: Prolonged dysfunction of the myocardium after a brief episode of severe ischemia, with gradual return of contractile activity.Heart Aneurysm: A localized bulging or dilatation in the muscle wall of a heart (MYOCARDIUM), usually in the LEFT VENTRICLE. Blood-filled aneurysms are dangerous because they may burst. Fibrous aneurysms interfere with the heart function through the loss of contractility. True aneurysm is bound by the vessel wall or cardiac wall. False aneurysms are HEMATOMA caused by myocardial rupture.Ventricular Septum: The muscular structure separating the right and the left lower chambers (HEART VENTRICLES) of the heart. The ventricular septum consists of a very small membranous portion just beneath the AORTIC VALVE, and a large thick muscular portion consisting of three sections including the inlet septum, the trabecular septum, and the outlet septum.Streptokinase: Streptococcal fibrinolysin . An enzyme produced by hemolytic streptococci. It hydrolyzes amide linkages and serves as an activator of plasminogen. It is used in thrombolytic therapy and is used also in mixtures with streptodornase (STREPTODORNASE AND STREPTOKINASE). EC 3.4.-.Blood Flow Velocity: A value equal to the total volume flow divided by the cross-sectional area of the vascular bed.Arrhythmias, Cardiac: Any disturbances of the normal rhythmic beating of the heart or MYOCARDIAL CONTRACTION. Cardiac arrhythmias can be classified by the abnormalities in HEART RATE, disorders of electrical impulse generation, or impulse conduction.Disease Models, Animal: Naturally occurring or experimentally induced animal diseases with pathological processes sufficiently similar to those of human diseases. They are used as study models for human diseases.Myocardial Revascularization: The restoration of blood supply to the myocardium. (From Dorland, 28th ed)Tissue Survival: The span of viability of a tissue or an organ.Pacemaker, Artificial: A device designed to stimulate, by electric impulses, contraction of the heart muscles. It may be temporary (external) or permanent (internal or internal-external).Heart Septum: This structure includes the thin muscular atrial septum between the two HEART ATRIA, and the thick muscular ventricular septum between the two HEART VENTRICLES.Heart Valve Prosthesis Implantation: Surgical insertion of synthetic material to repair injured or diseased heart valves.Observer Variation: The failure by the observer to measure or identify a phenomenon accurately, which results in an error. Sources for this may be due to the observer's missing an abnormality, or to faulty technique resulting in incorrect test measurement, or to misinterpretation of the data. Two varieties are inter-observer variation (the amount observers vary from one another when reporting on the same material) and intra-observer variation (the amount one observer varies between observations when reporting more than once on the same material).Metoprolol: A selective adrenergic beta-1 blocking agent that is commonly used to treat ANGINA PECTORIS; HYPERTENSION; and CARDIAC ARRHYTHMIAS.Endocardium: The innermost layer of the heart, comprised of endothelial cells.Cardiac Output, Low: A state of subnormal or depressed cardiac output at rest or during stress. It is a characteristic of CARDIOVASCULAR DISEASES, including congenital, valvular, rheumatic, hypertensive, coronary, and cardiomyopathic. The serious form of low cardiac output is characterized by marked reduction in STROKE VOLUME, and systemic vasoconstriction resulting in cold, pale, and sometimes cyanotic extremities.Exercise Tolerance: The exercise capacity of an individual as measured by endurance (maximal exercise duration and/or maximal attained work load) during an EXERCISE TEST.Heart Valve Prosthesis: A device that substitutes for a heart valve. It may be composed of biological material (BIOPROSTHESIS) and/or synthetic material.Myocarditis: Inflammatory processes of the muscular walls of the heart (MYOCARDIUM) which result in injury to the cardiac muscle cells (MYOCYTES, CARDIAC). Manifestations range from subclinical to sudden death (DEATH, SUDDEN). Myocarditis in association with cardiac dysfunction is classified as inflammatory CARDIOMYOPATHY usually caused by INFECTION, autoimmune diseases, or responses to toxic substances. Myocarditis is also a common cause of DILATED CARDIOMYOPATHY and other cardiomyopathies.Analysis of Variance: A statistical technique that isolates and assesses the contributions of categorical independent variables to variation in the mean of a continuous dependent variable.Heart Defects, Congenital: Developmental abnormalities involving structures of the heart. These defects are present at birth but may be discovered later in life.Myocytes, Cardiac: Striated muscle cells found in the heart. They are derived from cardiac myoblasts (MYOBLASTS, CARDIAC).Aortic Valve: The valve between the left ventricle and the ascending aorta which prevents backflow into the left ventricle.Hypertrophy, Right Ventricular: Enlargement of the RIGHT VENTRICLE of the heart. This increase in ventricular mass is often attributed to PULMONARY HYPERTENSION and is a contributor to cardiovascular morbidity and mortality.Chronic Disease: Diseases which have one or more of the following characteristics: they are permanent, leave residual disability, are caused by nonreversible pathological alteration, require special training of the patient for rehabilitation, or may be expected to require a long period of supervision, observation, or care. (Dictionary of Health Services Management, 2d ed)Atrial Fibrillation: Abnormal cardiac rhythm that is characterized by rapid, uncoordinated firing of electrical impulses in the upper chambers of the heart (HEART ATRIA). In such case, blood cannot be effectively pumped into the lower chambers of the heart (HEART VENTRICLES). It is caused by abnormal impulse generation.Reproducibility of Results: The statistical reproducibility of measurements (often in a clinical context), including the testing of instrumentation or techniques to obtain reproducible results. The concept includes reproducibility of physiological measurements, which may be used to develop rules to assess probability or prognosis, or response to a stimulus; reproducibility of occurrence of a condition; and reproducibility of experimental results.Coronary Artery Disease: Pathological processes of CORONARY ARTERIES that may derive from a congenital abnormality, atherosclerotic, or non-atherosclerotic cause.Pulmonary Valve Insufficiency: Backflow of blood from the PULMONARY ARTERY into the RIGHT VENTRICLE due to imperfect closure of the PULMONARY VALVE.Severity of Illness Index: Levels within a diagnostic group which are established by various measurement criteria applied to the seriousness of a patient's disorder.Hypertension, Pulmonary: Increased VASCULAR RESISTANCE in the PULMONARY CIRCULATION, usually secondary to HEART DISEASES or LUNG DISEASES.Thrombolytic Therapy: Use of infusions of FIBRINOLYTIC AGENTS to destroy or dissolve thrombi in blood vessels or bypass grafts.Electrocardiography, Ambulatory: Method in which prolonged electrocardiographic recordings are made on a portable tape recorder (Holter-type system) or solid-state device ("real-time" system), while the patient undergoes normal daily activities. It is useful in the diagnosis and management of intermittent cardiac arrhythmias and transient myocardial ischemia.Postoperative Period: The period following a surgical operation.Radiopharmaceuticals: Compounds that are used in medicine as sources of radiation for radiotherapy and for diagnostic purposes. They have numerous uses in research and industry. (Martindale, The Extra Pharmacopoeia, 30th ed, p1161)Physical Exertion: Expenditure of energy during PHYSICAL ACTIVITY. Intensity of exertion may be measured by rate of OXYGEN CONSUMPTION; HEAT produced, or HEART RATE. Perceived exertion, a psychological measure of exertion, is included.Propanolamines: AMINO ALCOHOLS containing the propanolamine (NH2CH2CHOHCH2) group and its derivatives.Vascular Resistance: The force that opposes the flow of BLOOD through a vascular bed. It is equal to the difference in BLOOD PRESSURE across the vascular bed divided by the CARDIAC OUTPUT.Heart Arrest, Induced: A procedure to stop the contraction of MYOCARDIUM during HEART SURGERY. It is usually achieved with the use of chemicals (CARDIOPLEGIC SOLUTIONS) or cold temperature (such as chilled perfusate).Cardiomegaly: Enlargement of the HEART, usually indicated by a cardiothoracic ratio above 0.50. Heart enlargement may involve the right, the left, or both HEART VENTRICLES or HEART ATRIA. Cardiomegaly is a nonspecific symptom seen in patients with chronic systolic heart failure (HEART FAILURE) or several forms of CARDIOMYOPATHIES.Retrospective Studies: Studies used to test etiologic hypotheses in which inferences about an exposure to putative causal factors are derived from data relating to characteristics of persons under study or to events or experiences in their past. The essential feature is that some of the persons under study have the disease or outcome of interest and their characteristics are compared with those of unaffected persons.Thallium Radioisotopes: Unstable isotopes of thallium that decay or disintegrate emitting radiation. Tl atoms with atomic weights 198-202, 204, and 206-210 are thallium radioisotopes.Heart-Assist Devices: Small pumps, often implantable, designed for temporarily assisting the heart, usually the LEFT VENTRICLE, to pump blood. They consist of a pumping chamber and a power source, which may be partially or totally external to the body and activated by electromagnetic motors.Cardiomyopathy, Hypertrophic: A form of CARDIAC MUSCLE disease, characterized by left and/or right ventricular hypertrophy (HYPERTROPHY, LEFT VENTRICULAR; HYPERTROPHY, RIGHT VENTRICULAR), frequent asymmetrical involvement of the HEART SEPTUM, and normal or reduced left ventricular volume. Risk factors include HYPERTENSION; AORTIC STENOSIS; and gene MUTATION; (FAMILIAL HYPERTROPHIC CARDIOMYOPATHY).Aortic Valve Insufficiency: Pathological condition characterized by the backflow of blood from the ASCENDING AORTA back into the LEFT VENTRICLE, leading to regurgitation. It is caused by diseases of the AORTIC VALVE or its surrounding tissue (aortic root).Magnetic Resonance Imaging: Non-invasive method of demonstrating internal anatomy based on the principle that atomic nuclei in a strong magnetic field absorb pulses of radiofrequency energy and emit them as radiowaves which can be reconstructed into computerized images. The concept includes proton spin tomographic techniques.Oxygen Consumption: The rate at which oxygen is used by a tissue; microliters of oxygen STPD used per milligram of tissue per hour; the rate at which oxygen enters the blood from alveolar gas, equal in the steady state to the consumption of oxygen by tissue metabolism throughout the body. (Stedman, 25th ed, p346)Hypoplastic Left Heart Syndrome: A condition caused by underdevelopment of the whole left half of the heart. It is characterized by hypoplasia of the left cardiac chambers (HEART ATRIUM; HEART VENTRICLE), the AORTA, the AORTIC VALVE, and the MITRAL VALVE. Severe symptoms appear in early infancy when DUCTUS ARTERIOSUS closes.Fibrosis: Any pathological condition where fibrous connective tissue invades any organ, usually as a consequence of inflammation or other injury.Postoperative Complications: Pathologic processes that affect patients after a surgical procedure. They may or may not be related to the disease for which the surgery was done, and they may or may not be direct results of the surgery.Collateral Circulation: Maintenance of blood flow to an organ despite obstruction of a principal vessel. Blood flow is maintained through small vessels.Trimetazidine: A vasodilator used in angina of effort or ischemic heart disease.Echocardiography, Transesophageal: Ultrasonic recording of the size, motion, and composition of the heart and surrounding tissues using a transducer placed in the esophagus.Pulmonary Wedge Pressure: The blood pressure as recorded after wedging a CATHETER in a small PULMONARY ARTERY; believed to reflect the PRESSURE in the pulmonary CAPILLARIES.Cardiovascular Agents: Agents that affect the rate or intensity of cardiac contraction, blood vessel diameter, or blood volume.Edema, Cardiac: Abnormal fluid retention by the body due to impaired cardiac function or heart failure. It is usually characterized by increase in venous and capillary pressure, and swollen legs when standing. It is different from the generalized edema caused by renal dysfunction (NEPHROTIC SYNDROME).Tachycardia, Ventricular: An abnormally rapid ventricular rhythm usually in excess of 150 beats per minute. It is generated within the ventricle below the BUNDLE OF HIS, either as autonomic impulse formation or reentrant impulse conduction. Depending on the etiology, onset of ventricular tachycardia can be paroxysmal (sudden) or nonparoxysmal, its wide QRS complexes can be uniform or polymorphic, and the ventricular beating may be independent of the atrial beating (AV dissociation).Feasibility Studies: Studies to determine the advantages or disadvantages, practicability, or capability of accomplishing a projected plan, study, or project.Echocardiography, Three-Dimensional: Echocardiography amplified by the addition of depth to the conventional two-dimensional ECHOCARDIOGRAPHY visualizing only the length and width of the heart. Three-dimensional ultrasound imaging was first described in 1961 but its application to echocardiography did not take place until 1974. (Mayo Clin Proc 1993;68:221-40)Angiotensin-Converting Enzyme Inhibitors: A class of drugs whose main indications are the treatment of hypertension and heart failure. They exert their hemodynamic effect mainly by inhibiting the renin-angiotensin system. They also modulate sympathetic nervous system activity and increase prostaglandin synthesis. They cause mainly vasodilation and mild natriuresis without affecting heart rate and contractility.Contrast Media: Substances used to allow enhanced visualization of tissues.Pericardium: A conical fibro-serous sac surrounding the HEART and the roots of the great vessels (AORTA; VENAE CAVAE; PULMONARY ARTERY). Pericardium consists of two sacs: the outer fibrous pericardium and the inner serous pericardium. The latter consists of an outer parietal layer facing the fibrous pericardium, and an inner visceral layer (epicardium) resting next to the heart, and a pericardial cavity between these two layers.Fontan Procedure: A procedure in which total right atrial or total caval blood flow is channeled directly into the pulmonary artery or into a small right ventricle that serves only as a conduit. The principal congenital malformations for which this operation is useful are TRICUSPID ATRESIA and single ventricle with pulmonary stenosis.Risk Factors: An aspect of personal behavior or lifestyle, environmental exposure, or inborn or inherited characteristic, which, on the basis of epidemiologic evidence, is known to be associated with a health-related condition considered important to prevent.Organ Size: The measurement of an organ in volume, mass, or heaviness.Rest: Freedom from activity.Heart Valve Diseases: Pathological conditions involving any of the various HEART VALVES and the associated structures (PAPILLARY MUSCLES and CHORDAE TENDINEAE).Anterior Wall Myocardial Infarction: MYOCARDIAL INFARCTION in which the anterior wall of the heart is involved. Anterior wall myocardial infarction is often caused by occlusion of the left anterior descending coronary artery. It can be categorized as anteroseptal or anterolateral wall myocardial infarction.Cardiopulmonary Bypass: Diversion of the flow of blood from the entrance of the right atrium directly to the aorta (or femoral artery) via an oxygenator thus bypassing both the heart and lungs.Heart Block: Impaired conduction of cardiac impulse that can occur anywhere along the conduction pathway, such as between the SINOATRIAL NODE and the right atrium (SA block) or between atria and ventricles (AV block). Heart blocks can be classified by the duration, frequency, or completeness of conduction block. Reversibility depends on the degree of structural or functional defects.Survival Rate: The proportion of survivors in a group, e.g., of patients, studied and followed over a period, or the proportion of persons in a specified group alive at the beginning of a time interval who survive to the end of the interval. It is often studied using life table methods.Random Allocation: A process involving chance used in therapeutic trials or other research endeavor for allocating experimental subjects, human or animal, between treatment and control groups, or among treatment groups. It may also apply to experiments on inanimate objects.Sensitivity and Specificity: Binary classification measures to assess test results. Sensitivity or recall rate is the proportion of true positives. Specificity is the probability of correctly determining the absence of a condition. (From Last, Dictionary of Epidemiology, 2d ed)Linear Models: Statistical models in which the value of a parameter for a given value of a factor is assumed to be equal to a + bx, where a and b are constants. The models predict a linear regression.Cardiovascular Surgical Procedures: Surgery performed on the heart or blood vessels.Coronary Vessel Anomalies: Malformations of CORONARY VESSELS, either arteries or veins. Included are anomalous origins of coronary arteries; ARTERIOVENOUS FISTULA; CORONARY ANEURYSM; MYOCARDIAL BRIDGING; and others.Myocardial Perfusion Imaging: The creation and display of functional images showing where the blood is flowing into the MYOCARDIUM by following over time the distribution of tracers injected into the blood stream.Carbazoles: Benzo-indoles similar to CARBOLINES which are pyrido-indoles. In plants, carbazoles are derived from indole and form some of the INDOLE ALKALOIDS.Heart Transplantation: The transference of a heart from one human or animal to another.IodobenzenesImage Processing, Computer-Assisted: A technique of inputting two-dimensional images into a computer and then enhancing or analyzing the imagery into a form that is more useful to the human observer.Tachycardia: Abnormally rapid heartbeat, usually with a HEART RATE above 100 beats per minute for adults. Tachycardia accompanied by disturbance in the cardiac depolarization (cardiac arrhythmia) is called tachyarrhythmia.Heart Sounds: The sounds heard over the cardiac region produced by the functioning of the heart. There are four distinct sounds: the first occurs at the beginning of SYSTOLE and is heard as a "lubb" sound; the second is produced by the closing of the AORTIC VALVE and PULMONARY VALVE and is heard as a "dupp" sound; the third is produced by vibrations of the ventricular walls when suddenly distended by the rush of blood from the HEART ATRIA; and the fourth is produced by atrial contraction and ventricular filling.Tomography, Emission-Computed: Tomography using radioactive emissions from injected RADIONUCLIDES and computer ALGORITHMS to reconstruct an image.Bundle-Branch Block: A form of heart block in which the electrical stimulation of HEART VENTRICLES is interrupted at either one of the branches of BUNDLE OF HIS thus preventing the simultaneous depolarization of the two ventricles.Double-Blind Method: A method of studying a drug or procedure in which both the subjects and investigators are kept unaware of who is actually getting which specific treatment.Case-Control Studies: Studies which start with the identification of persons with a disease of interest and a control (comparison, referent) group without the disease. The relationship of an attribute to the disease is examined by comparing diseased and non-diseased persons with regard to the frequency or levels of the attribute in each group.Tantalum: Tantalum. A rare metallic element, atomic number 73, atomic weight 180.948, symbol Ta. It is a noncorrosive and malleable metal that has been used for plates or disks to replace cranial defects, for wire sutures, and for making prosthetic devices. (Dorland, 28th ed)Organotechnetium Compounds: Organic compounds that contain technetium as an integral part of the molecule. These compounds are often used as radionuclide imaging agents.Hypertension: Persistently high systemic arterial BLOOD PRESSURE. Based on multiple readings (BLOOD PRESSURE DETERMINATION), hypertension is currently defined as when SYSTOLIC PRESSURE is consistently greater than 140 mm Hg or when DIASTOLIC PRESSURE is consistently 90 mm Hg or more.Defibrillators, Implantable: Implantable devices which continuously monitor the electrical activity of the heart and automatically detect and terminate ventricular tachycardia (TACHYCARDIA, VENTRICULAR) and VENTRICULAR FIBRILLATION. They consist of an impulse generator, batteries, and electrodes.Biological Markers: Measurable and quantifiable biological parameters (e.g., specific enzyme concentration, specific hormone concentration, specific gene phenotype distribution in a population, presence of biological substances) which serve as indices for health- and physiology-related assessments, such as disease risk, psychiatric disorders, environmental exposure and its effects, disease diagnosis, metabolic processes, substance abuse, pregnancy, cell line development, epidemiologic studies, etc.Tomography, X-Ray Computed: Tomography using x-ray transmission and a computer algorithm to reconstruct the image.Pericardial Effusion: Fluid accumulation within the PERICARDIUM. Serous effusions are associated with pericardial diseases. Hemopericardium is associated with trauma. Lipid-containing effusion (chylopericardium) results from leakage of THORACIC DUCT. Severe cases can lead to CARDIAC TAMPONADE.Cardioplegic Solutions: Solutions which, upon administration, will temporarily arrest cardiac activity. They are used in the performance of heart surgery.Rats, Wistar: A strain of albino rat developed at the Wistar Institute that has spread widely at other institutions. This has markedly diluted the original strain.Pressure: A type of stress exerted uniformly in all directions. Its measure is the force exerted per unit area. (McGraw-Hill Dictionary of Scientific and Technical Terms, 6th ed)Adrenergic beta-Agonists: Drugs that selectively bind to and activate beta-adrenergic receptors.Mitral Valve Stenosis: Narrowing of the passage through the MITRAL VALVE due to FIBROSIS, and CALCINOSIS in the leaflets and chordal areas. This elevates the left atrial pressure which, in turn, raises pulmonary venous and capillary pressure leading to bouts of DYSPNEA and TACHYCARDIA during physical exertion. RHEUMATIC FEVER is its primary cause.Atrial Natriuretic Factor: A potent natriuretic and vasodilatory peptide or mixture of different-sized low molecular weight PEPTIDES derived from a common precursor and secreted mainly by the HEART ATRIUM. All these peptides share a sequence of about 20 AMINO ACIDS.Tricuspid Valve Insufficiency: Backflow of blood from the RIGHT VENTRICLE into the RIGHT ATRIUM due to imperfect closure of the TRICUSPID VALVE.Endocardial Fibroelastosis: A condition characterized by the thickening of ENDOCARDIUM due to proliferation of fibrous and elastic tissue, usually in the left ventricle leading to impaired cardiac function (CARDIOMYOPATHY, RESTRICTIVE). It is most commonly seen in young children and rarely in adults. It is often associated with congenital heart anomalies (HEART DEFECTS CONGENITAL;) INFECTION; or gene mutation. Defects in the tafazzin protein, encoded by TAZ gene, result in a form of autosomal dominant familial endocardial fibroelastosis.Adrenergic beta-1 Receptor Agonists: Compounds that bind to and activate ADRENERGIC BETA-1 RECEPTORS.Rats, Sprague-Dawley: A strain of albino rat used widely for experimental purposes because of its calmness and ease of handling. It was developed by the Sprague-Dawley Animal Company.Perfusion: Treatment process involving the injection of fluid into an organ or tissue.Takotsubo Cardiomyopathy: A transient left ventricular apical dysfunction or ballooning accompanied by electrocardiographic (ECG) T wave inversions. This abnormality is associated with high levels of CATECHOLAMINES, either administered or endogenously secreted from a tumor or during extreme stress.Anti-Arrhythmia Agents: Agents used for the treatment or prevention of cardiac arrhythmias. They may affect the polarization-repolarization phase of the action potential, its excitability or refractoriness, or impulse conduction or membrane responsiveness within cardiac fibers. Anti-arrhythmia agents are often classed into four main groups according to their mechanism of action: sodium channel blockade, beta-adrenergic blockade, repolarization prolongation, or calcium channel blockade.Vasodilator Agents: Drugs used to cause dilation of the blood vessels.Acute Disease: Disease having a short and relatively severe course.Risk Assessment: The qualitative or quantitative estimation of the likelihood of adverse effects that may result from exposure to specified health hazards or from the absence of beneficial influences. (Last, Dictionary of Epidemiology, 1988)Swine: Any of various animals that constitute the family Suidae and comprise stout-bodied, short-legged omnivorous mammals with thick skin, usually covered with coarse bristles, a rather long mobile snout, and small tail. Included are the genera Babyrousa, Phacochoerus (wart hogs), and Sus, the latter containing the domestic pig (see SUS SCROFA).Intra-Aortic Balloon Pumping: Counterpulsation in which a pumping unit synchronized with the patient's electrocardiogram rapidly fills a balloon in the aorta with helium or carbon dioxide in early diastole and evacuates the balloon at the onset of systole. As the balloon inflates, it raises aortic diastolic pressure, and as it deflates, it lowers aortic systolic pressure. The result is a decrease in left ventricular work and increased myocardial and peripheral perfusion.Coronary Stenosis: Narrowing or constriction of a coronary artery.Statistics, Nonparametric: A class of statistical methods applicable to a large set of probability distributions used to test for correlation, location, independence, etc. In most nonparametric statistical tests, the original scores or observations are replaced by another variable containing less information. An important class of nonparametric tests employs the ordinal properties of the data. Another class of tests uses information about whether an observation is above or below some fixed value such as the median, and a third class is based on the frequency of the occurrence of runs in the data. (From McGraw-Hill Dictionary of Scientific and Technical Terms, 4th ed, p1284; Corsini, Concise Encyclopedia of Psychology, 1987, p764-5)Models, Cardiovascular: Theoretical representations that simulate the behavior or activity of the cardiovascular system, processes, or phenomena; includes the use of mathematical equations, computers and other electronic equipment.Extracorporeal Circulation: Diversion of blood flow through a circuit located outside the body but continuous with the bodily circulation.Scintillation Counting: Detection and counting of scintillations produced in a fluorescent material by ionizing radiation.Chi-Square Distribution: A distribution in which a variable is distributed like the sum of the squares of any given independent random variable, each of which has a normal distribution with mean of zero and variance of one. The chi-square test is a statistical test based on comparison of a test statistic to a chi-square distribution. The oldest of these tests are used to detect whether two or more population distributions differ from one another.Angiography: Radiography of blood vessels after injection of a contrast medium.Technetium: The first artificially produced element and a radioactive fission product of URANIUM. Technetium has the atomic symbol Tc, atomic number 43, and atomic weight 98.91. All technetium isotopes are radioactive. Technetium 99m (m=metastable) which is the decay product of Molybdenum 99, has a half-life of about 6 hours and is used diagnostically as a radioactive imaging agent. Technetium 99 which is a decay product of technetium 99m, has a half-life of 210,000 years.

Myocardial oxygenation during high work states in hearts with postinfarction remodeling. (1/6555)

BACKGROUND: Postinfarction left ventricular remodeling (LVR) is associated with reductions in myocardial high-energy phosphate (HEP) levels, which are more severe in animals that develop overt congestive heart failure (CHF). During high work states, further HEP loss occurs, which suggests demand-induced ischemia. This study tested the hypothesis that inadequate myocyte oxygen availability is the basis for these HEP abnormalities. METHODS AND RESULTS: Myocardial infarction was produced by left circumflex coronary artery ligation in swine. Studies were performed in 20 normal animals, 14 animals with compensated LVR, and 9 animals with CHF. Phosphocreatine (PCr)/ATP was determined with 31P NMR and deoxymyoglobin (Mb-delta) with 1H NMR in myocardium remote from the infarct. Basal PCr/ATP tended to be decreased in postinfarct hearts, and this was significant in animals with CHF. Infusion of dobutamine (20 microg x kg-1 x min-1 IV) caused doubling of the rate-pressure product in both normal and LVR hearts and resulted in comparable significant decreases of PCr/ATP in both groups. This decrease in PCr/ATP was not associated with detectable Mb-delta. In CHF hearts, rate-pressure product increased only 40% in response to dobutamine; this attenuated response also was not associated with detectable Mb-delta. CONCLUSIONS: Thus, the decrease of PCr/ATP during dobutamine infusion is not the result of insufficient myocardial oxygen availability. Furthermore, in CHF hearts, the low basal PCr/ATP and the attenuated response to dobutamine occurred in the absence of myocardial hypoxia, indicating that the HEP and contractile abnormalities were not the result of insufficient oxygen availability.  (+info)

Hemodialysis with high-calcium dialysate impairs cardiac relaxation. (2/6555)

BACKGROUND: During hemodialysis (HD), serum ionized calcium is directly related to the dialysate calcium concentration. We have recently shown an acute induction of hypercalcemia to impair left ventricular (LV) relaxation. In the current study we sought to establish whether changes in serum Ca++ also affect LV function during HD. METHODS: We echocardiographically examined the LV relaxation and systolic function of 12 patients with end-stage renal disease before and after three HD treatments with dialysate Ca++ concentrations of 1.25 mmol/liter (dCa++1.25), 1.5 mmol/liter (dCa++1.50), and 1.75 mmol/liter (dCa++1.75), respectively. Age- and sex-matched healthy controls were also examined echocardiographically. RESULTS: The LV posterior wall thickness and the interventricular septum thickness, and the LV end-diastolic dimension and the end-systolic dimensions were significantly greater in the patients when compared with the controls, and the LV fractional shortening, the ratio of peak early to peak late diastolic velocities (E/Amax), and the isovolumic relaxation time (IVRT) showed impairment of LV relaxation and systolic function in the patients. Serum ionized calcium increased significantly during the dCa++1.5 HD (1.24 +/- 0.10 vs. 1.34 +/- 0.06 mmol/liter, P = 0. 004) and dCa++1.75 HD (1.19 +/- 0.10 vs. 1.47 +/- 0.06 mmol/liter, P = 0.002), and plasma intact parathyroid hormone decreased significantly during the dCa++1.75 HD (medians 8.2 vs. 2.7 pmol/liter, P = 0.002). LV systolic function was not altered during any of the treatments. The changes in E/Amax and IVRT suggested impairment of relaxation during all sessions, but only during the dCa++1.75 HD was the impairment statistically significant (E/Amax 1. 153 +/- 0.437 vs. 0.943 +/- 0.352, P < 0.05; IVRT 147 +/- 29 vs. 175 +/- 50 msecond, P < 0.05). CONCLUSION: HD with high-calcium (dCa++1. 75 mmol/liter) dialysate impairs LV relaxation when compared with lower calcium dialysate (dCa++1.25 and dCa++1.5 mmol/liter) treatments.  (+info)

A comparison of an A1 adenosine receptor agonist (CVT-510) with diltiazem for slowing of AV nodal conduction in guinea-pig. (3/6555)

1. The purpose of this study was to compare the pharmacological properties (i.e. the AV nodal depressant, vasodilator, and inotropic effects) of two AV nodal blocking agents belonging to different drug classes; a novel A1 adenosine receptor (A1 receptor) agonist, N-(3(R)-tetrahydrofuranyl)-6-aminopurine riboside (CVT-510), and the prototypical calcium channel blocker diltiazem. 2. In the atrial-paced isolated heart, CVT-510 was approximately 5 fold more potent to prolong the stimulus-to-His bundle (S-H interval), a measure of slowing AV nodal conduction (EC50 = 41 nM) than to increase coronary conductance (EC50 = 200 nM). At concentrations of CVT-510 (40 nM) and diltiazem (1 microM) that caused equal prolongation of S-H interval (approximately 10 ms), diltiazem, but not CVT-510, significantly reduced left ventricular developed pressure (LVP) and markedly increased coronary conductance. CVT-510 shortened atrial (EC50 = 73 nM) but not the ventricular monophasic action potentials (MAP). 3. In atrial-paced anaesthetized guinea-pigs, intravenous infusions of CVT-510 and diltiazem caused nearly equal prolongations of P-R interval. However, diltiazem, but not CVT-510, significantly reduced mean arterial blood pressure. 4. Both CVT-510 and diltiazem prolonged S-H interval, i.e., slowed AV nodal conduction. However, the A1 receptor-selective agonist CVT-510 did so without causing the negative inotropic, vasodilator, and hypotensive effects associated with diltiazem. Because CVT-510 did not affect the ventricular action potential, it is unlikely that this agonist will have a proarrythmic action in ventricular myocardium.  (+info)

Effects of tumour necrosis factor-alpha on left ventricular function in the rat isolated perfused heart: possible mechanisms for a decline in cardiac function. (4/6555)

1. The cardiac depressant actions of TNF were investigated in the isolated perfused rat heart under constant flow (10 ml min(-1)) and constant pressure (70 mmHg) conditions, using a recirculating (50 ml) mode of perfusion. 2. Under constant flow conditions TNF (20 ng ml(-1)) caused an early (< 25 min) decrease in left ventricular developed pressure (LVDP), which was maintained for 90 min (LVDP after 90 min: control vs TNF; 110 +/- 4 vs 82 +/- 10 mmHg, P < 0.01). 3. The depression in cardiac function seen with TNF under constant flow conditions, was blocked by the ceramidase inhibitor N-oleoylethanolamine (NOE), 1 microM, (LVDP after 90 min: TNF vs TNF with NOE; 82 +/- 10 vs 11 +/- 5 mmHg, P < 0.05). 4. In hearts perfused at constant pressure, TNF caused a decrease in coronary flow rate (change in flow 20 min after TNF: control vs TNF; -3.0 +/- 0.9 vs -8.7 +/- 1.2 ml min(-1), P < 0.01). This was paralleled by a negative inotropic effect (change in LVDP 20 min after TNF: control vs TNF; -17 +/- 7 vs -46 +/- 6 mmHg, P < 0.01). The decline in function was more rapid and more severe than that seen under conditions of constant flow. 5. These data indicate that cardiac function can be disrupted by TNF on two levels, firstly via a direct, ceramidase dependant negative inotropic effect, and secondly via an indirect coronary vasoconstriction.  (+info)

In vivo demonstration of H3-histaminergic inhibition of cardiac sympathetic stimulation by R-alpha-methyl-histamine and its prodrug BP 2.94 in the dog. (5/6555)

1. The aim of this study was to investigate whether histamine H3-receptor agonists could inhibit the effects of cardiac sympathetic nerve stimulation in the dog. 2. Catecholamine release by the heart and the associated variation of haemodynamic parameters were measured after electrical stimulation of the right cardiac sympathetic nerves (1-4 Hz, 10 V, 10 ms) in the anaesthetized dog treated with R-alpha-methyl-histamine (R-HA) and its prodrug BP 2.94 (BP). 3. Cardiac sympathetic stimulation induced a noradrenaline release into the coronary sinus along with a tachycardia and an increase in left ventricular pressure and contractility without changes in mean arterial pressure. Intravenous administration of H3-receptor agonists significantly decreased noradrenaline release by the heart (R-HA at 2 micromol kg(-1) h(-1): +77 +/- 25 vs +405 +/- 82; BP 2.94 at 1 mg kg(-1): +12 +/- 11 vs +330 +/- 100 pg ml(-1) in control conditions, P < or = 0.05), and increases in heart rate (R-HA at 2 micromol kg(-1) h(-1): +26 +/- 8 vs +65 +/- 10 and BP 2.94 at 1 mg kg(-1): +30 +/- 8 vs 75 +/- 6 beats min(-1), in control conditions P < or = 0.05), left ventricular pressure, and contractility. Treatment with SC 359 (1 mg kg(-1)) a selective H3-antagonist, reversed the effects of H3-receptor agonists. Treatment with R-HA at 2 micromol kg(-1) h(-1) and BP 2.94 at 1 mg kg(-1) tended to decrease, while that with SC 359 significantly increased basal heart rate (from 111 +/- 3 to 130 +/- 5 beats min(-1), P < or = 0.001). 4. Functional H3-receptors are present on sympathetic nerve endings in the dog heart. Their stimulation by R-alpha-methyl-histamine or BP 2.94 can inhibit noradrenaline release by the heart and its associated haemodynamic effects.  (+info)

Impact of vascular adaptation to chronic aortic regurgitation on left ventricular performance. (6/6555)

BACKGROUND: This investigation was designed to test the hypothesis that vascular adaptation occurs in patients with chronic aortic regurgitation to maintain left ventricular (LV) performance. METHODS AND RESULTS: Forty-five patients with chronic aortic regurgitation (mean age 50+/-14 years) were studied using a micromanometer LV catheter to obtain LV pressures and radionuclide ventriculography to obtain LV volumes during multiple loading conditions and right atrial pacing. These 45 patients were subgrouped according to their LV contractility (Ees) and ejection fraction values. Group I consisted of 24 patients with a normal Ees. Group IIa consisted of 10 patients with impaired Ees values (Ees <1.00 mm Hg/mL) but normal LV ejection fractions; Group IIb consisted of 11 patients with impaired contractility and reduced LV ejection fractions. The left ventricular-arterial coupling ratio, Ees/Ea, where Ea was calculated by dividing the LV end-systolic pressure by LV stroke volume, averaged 1.60+/-0.91 in Group I. It decreased to 0.91+/-0.27 in Group IIa (P<0.05 versus Group I), and it decreased further in Group IIb to 0.43+/-0.24 (P<0.001 versus Groups I and IIa). The LV ejection fractions were inversely related to the Ea values in both the normal and impaired contractility groups (r=-0.48, P<0.05 and r=-0.56, P<0.01, respectively), although the slopes of these relationships differed (P<0.05). The average LV work was maximal in Group IIa when the left ventricular-arterial coupling ratio was near 1.0 because of a significant decrease in total arterial elastance (P<0.01 versus Group I). In contrast, the decrease in the left ventricular-arterial coupling ratio in Group IIb was caused by an increase in total arterial elastance, effectively double loading the LV, contributing to a decrease in LV pump efficiency (P<0.01 versus Group IIa and P<0.001 versus Group I). CONCLUSIONS: Vascular adaptation may be heterogeneous in patients with chronic aortic regurgitation. In some, total arterial elastance decreases to maximize LV work and maintain LV performance, whereas in others, it increases, thereby double loading the LV, contributing to afterload excess and a deterioration in LV performance that is most prominent in those with impaired contractility.  (+info)

Studies on structural changes of the carotid arteries and the heart in asymptomatic renal transplant recipients. (7/6555)

BACKGROUND: The present study was designed to characterize early structural changes of large arteries in renal transplant recipients with no clinical evidence of cardiovascular disease and normal blood pressure values, and to analyse the relationship between arterial alterations and those of the heart. METHODS: Intima media thickness and atherosclerotic plaques of the carotid arteries as well as left ventricular geometry and function were examined in 35 asymtomatic renal transplant recipients and 29 age- and sex-matched healthy controls by high resolution B-mode ultrasound and by echocardiography. RESULTS: Intima-media thickness of the carotid arteries was significantly higher in renal transplant recipients (1.21+/-0.08 mm) than in healthy controls (0.74+/-0.04 mm) (P<0.001). Atherosclerotic plaques were found in the majority of renal transplant recipients (71% vs 14% in healthy controls, P<0.001). Left ventricular mass index was significantly increased in the group of renal transplant recipients (264+/-13 g, 146+/-7 g/m2) when compared with healthy controls (155+/-8 g, 83+/-4 g/m2) (P<0.001). Multiple regression analysis in renal transplant recipients showed that intima media thickness of the carotid arteries was significantly related to left ventricular mass index (P<0.02), but not to age, blood pressure, body mass index, serum creatinine, cholesterol and lipoprotein (a) levels. In the group of healthy controls, intima-media thickness of the carotid artery was related to age (P<0.002), but not to left ventricular mass index or the other independent variables. CONCLUSIONS: The present study documents pronounced intima-media thickening in asymptomatic renal transplant recipients. Atherosclerotic lesions are present in most renal transplant recipients with no clinical evidence of cardiovascular disease. We observed a parallelism between arterial wall thickening and left ventricular hypertrophy, although blood pressure levels were normal during haemodialysis therapy and after renal transplantation.  (+info)

Effects of AT1 receptor blockade after myocardial infarct on myocardial fibrosis, stiffness, and contractility. (8/6555)

Angiotensin II type 1 (AT1) receptor blockade attenuates myocardial fibrosis after myocardial infarction (MI). However, whether inhibition of fibrosis by AT1 receptor blockade influences myocardial stiffness and contractility is unknown. We measured left ventricular (LV) hemodynamics, papillary muscle function, and myocardial stiffness and fibrosis in rats randomized to losartan or placebo 1 day after MI and treated subsequently for 8 wk. Losartan decreased LV and right ventricular weights as well as mean aortic and LV systolic pressures in sham and MI rats. LV end-diastolic pressure increased after MI and was decreased with losartan. Maximal developed tension and peak rate of tension rise and decline were decreased in MI vs. sham rats. Interstitial fibrosis developed after MI and was prevented in losartan-treated MI rats. The development of abnormal myocardial stiffness after MI was prevented by losartan. After MI, AT1 receptor blockade prevents an abnormal increase in myocardial collagen content. This effect was associated with a normalization of passive myocardial stiffness.  (+info)

Effect of ischemic arrest on left ventricular relaxation, diastolic stiffness and contractility<...Effect of ischemic arrest on left ventricular relaxation, diastolic stiffness and contractility<...
TY - JOUR. T1 - Effect of ischemic arrest on left ventricular relaxation, diastolic stiffness and contractility. AU - Schaff, Hartzell V. AU - Flaherty, J. T.. AU - Fredricksen, J. W.. PY - 1977. Y1 - 1977. N2 - The effect of ischemic arrest (IA) on ventricular relaxation and the diastolic pressure-volume (P/V) relationship was studied in 17 isolated, perfused, isovolumic feline hearts. During one hour of IA, nine hearts were maintained at normothermia (NIA) and eight were cooled to 27°C hypothermia (HIA). Maximum dP/dt was used as an index of contractility. P/V curves were inscribed by constant volume infusion into an intraventricular balloon and plotted according to the equation Pd = βe (dVd), where Pd = end diastolic pressure; Vd = end diastolic volume; (stiffnes constant) = slope of in Pvs Vd; and β = InP intercept at Vd = 0. To normalize for initial cavity size, curves were replotted as Pd vs (Vd-Vo)/V0, where Vo = volume at Pd of 10 mm Hg. Left ventricular relaxation was assessed by ...
Lower Myocardial Perfusion Reserve Is Associated With Decreased Regional Left Ventricular Function in Asymptomatic Participants...Lower Myocardial Perfusion Reserve Is Associated With Decreased Regional Left Ventricular Function in Asymptomatic Participants...
This study demonstrates a direct association between decreased regional LV function and reduced regional myocardial perfusion reserve in individuals with coronary artery disease risk factors but who are asymptomatic and have no history of heart disease. This relation was most pronounced in the inferior and posterior walls of the left ventricle. The association between perfusion and regional function was presumably weakened by confounding factors that could not be controlled in this population-based study, yet the observed relation was significant. The ability to detect such a subtle relation is attributed to the accuracy of MRI in quantifying both regional LV function and perfusion reserve.. Importantly, there was no relation between MBF and function at rest among MESA participants, whereas a significant association between regional function at rest with perfusion during adenosine induced hyperemia is documented. Therefore, our findings cannot be explained by local physiological mechanisms ...
Abstract 3688: Predictors of Contractile Recovery of Left Ventricular Function After Intracoronary Infusion of Bone-Marrow...Abstract 3688: Predictors of Contractile Recovery of Left Ventricular Function After Intracoronary Infusion of Bone-Marrow...
Background: The REPAIR-AMI trial has demonstrated that intracoronary infusion of bone marrow-derived progenitor cells (BMC) is associated with a larger recovery of left ventricular contractile function after acute myocardial infarction (AMI) compared to placebo. The present exploratory analysis aims to identify clinical and baseline LV functional predictors of the therapeutic benefit of intracoronary BMC infusion.. Methods: Using a double-blind, placebo controlled multicenter trial design, we randomized 204 patients with successfully reperfused acute myocardial infarction to receive intracoronary infusion of bone marrow - derived progenitor cells (BMC, n = 101) or placebo medium (n= 103) into the reperfused infarct artery. Primary end point was improvement of LV ejection fraction after 4 months.. Results: Overall, recovery of global left ventricular ejection fraction was significantly (p = 0.014) greater in the bone marrow-derived progenitor cell infusion group compared to placebo (absolute ...
PRIME PubMed | Association of Abnormal Left Ventricular Functional Reserve With Outcome in Heart Failure With Preserved...PRIME PubMed | Association of Abnormal Left Ventricular Functional Reserve With Outcome in Heart Failure With Preserved...
PubMed journal article: Association of Abnormal Left Ventricular Functional Reserve With Outcome in Heart Failure With Preserved Ejection Fraction. Download Prime PubMed App to iPhone, iPad, or Android
北京大学医学部机构知识库(IR@PKUHSC): INFLUENCE OF ABNORMAL FASTING PLASMA GLUCOSE ON LEFT VENTRICULAR FUNCTION IN OLDER PATIENTS WITH ACUTE...北京大学医学部机构知识库([email protected]): INFLUENCE OF ABNORMAL FASTING PLASMA GLUCOSE ON LEFT VENTRICULAR FUNCTION IN OLDER PATIENTS WITH ACUTE...
Yang Shiwei,Zhou Yujie,Nie Xiaomin,et al. INFLUENCE OF ABNORMAL FASTING PLASMA GLUCOSE ON LEFT VENTRICULAR FUNCTION IN OLDER PATIENTS WITH ACUTE MYOCARDIAL INFARCTION[J]. HEART,2011,suppl.3 ...
Chasing the Elusive Pressure-Volume Relationships | JACC: Cardiovascular ImagingChasing the Elusive Pressure-Volume Relationships | JACC: Cardiovascular Imaging
Quantification of ventricular pump function is fundamental to the practice of cardiology and is required for research into most aspects of cardiac physiology, disease, and therapeutics. Drawing an analogy between the heart and a steam engine, Otto Frank (1) introduced the pressure-volume diagram as a means of characterizing left ventricular properties in the 1890s. Nearly 80 years later, Suga (2) formalized the idea that the relationship between pressure and volume at end systole was relatively independent of loading conditions and that the slope of the curve, called Emax or Ees, was a sensitive measure of contractility. At nearly the same time, several investigators demonstrated how the relationship between pressure and volume at end diastole similarly provided the framework for quantifying passive properties of the myocardium (3,4). In the decades to follow, there was intensive research into the characteristics of the end-systolic pressure-volume relation (ESPVR) and end-diastolic ...
Ltered left ventricular function. This observation might be explained by the | IDH Inhibitor idhinhibitor.comLtered left ventricular function. This observation might be explained by the | IDH Inhibitor idhinhibitor.com
Ltered left ventricular function. This observation might be explained by the finding of Condorelli et al. emphasizing that a mild activation of Akt through
The NO-cGMP pathway participates in vascular adaptation to oxidative stress in Nrf2 KO mice | BMC Pharmacology and Toxicology |...The NO-cGMP pathway participates in vascular adaptation to oxidative stress in Nrf2 KO mice | BMC Pharmacology and Toxicology |...
We found that Nrf2 KO mice show an impaired left ventricular diastolic function as assessed by high resolution ultrasound. Accordingly, isolated perfused Nrf2 KO hearts showed an impaired response to β adrenergic stimulation by isoproterenol, while systolic left ventricular function was preserved. Surprisingly, blood pressure in Nrf2 KO mice was significantly decreased, and endothelial function of arterial conductance and coronary resistive vessels was preserved. This is consistent with an increased maximal dilation after vascular occlusion of the arteria iliaca externa, which indicates a fully preserved vascular and endothelial function in these mice. Mice lacking the endothelial nitric oxide synthase (eNOS KO) showed no dilatatory response to shear stress, confirming that flow-mediated-dilation response mainly depends on eNOS-dependent vasodilatory pathways. The circulating NO pool analysed by HPLC and chemiluminescence showed no differences between Nrf2 KO mice and WT littermates. However, ...
Heterogeneity of ventricular function and myocardial oxidative metabolism in nonischemic dilated cardiomyopathy | JACC: Journal...Heterogeneity of ventricular function and myocardial oxidative metabolism in nonischemic dilated cardiomyopathy | JACC: Journal...
Background. Heterogeneity in regional left ventricular function has long been noted in patients with nonischemic dilated cardiomyopathy. Regional variation in wall stress has been proposed as the pathophysiologic mechanism. By correlating regional function with oxidative metabolism, one can test the hypothesis that heterogeneity in wall stress is responsible for heterogeneity in function. We hypothesized that preserved function as a result of more favorable loading conditions would be associated with regional oxidative metabolism that is equal to or lower than that in other regions. ...
Effect of electrical activation site on left ventricular perfo...Effect of electrical activation site on left ventricular perfo...
Effect of electrical activation site on left ventricular performance in ventricular tachycardia patients with coronary heart disease.: Some patients with ventri
Iowa Heart First to Purchase Toshiba Aplio Artida with 3D Wall Motion Tracking | Imaging Technology NewsIowa Heart First to Purchase Toshiba Aplio Artida with 3D Wall Motion Tracking | Imaging Technology News
August 6, 2008 - Toshiba America Medical Systems Inc. delivered the first Aplio Artida ultrasound system to Iowa Heart Center in Des Moines. "Being the first to purchase the Aplio Artida is an exciting development for us and our patients," said Dr. Richard Marcus, director of cardiovascular ultrasound laboratory, Iowa Heart Center. "Having first-hand experience with the system, I am pleased with the images it produces. Its enhanced applications, particularly the 4D imaging, will allow our facility to continue to improve the care provided to our patients.". Dr. Marcus said the Iowa Heart Center selected the Aplio Artida for the systems impressive 4D imaging capability and 3D wall motion tracking.. Toshiba said Artidas real-time, multi-planar reformatting capabilities enable physicians to quantify global and regional LV function, including volumetric LV ejection fraction. Arbitrary views of the heart not available in 2D imaging are also obtained that can help with surgical planning.. The Artida ...
US7527599B2 - Method of determining cardiac indicators - Google PatentsUS7527599B2 - Method of determining cardiac indicators - Google Patents
A system and method for determining indicators of left ventricular function, and for determining the effectiveness of a cardiac therapy are disclosed. The indicators of left ventricular function can include isovolumetric relaxation time and the negative slope of a V wave obtained from measuring changes in left atrial pressure during diastole. The apparatus may include a catheter comprising an inflatable balloon for insertion into the esophagus of an individual, a compressor device for pressurizing the balloon, and a means for sensing and recording pressure changes on the balloon.
Role of Revascularization to Improve Left Ventricular Function | Color Atlas and Synopsis of Heart Failure | AccessCardiology |...Role of Revascularization to Improve Left Ventricular Function | Color Atlas and Synopsis of Heart Failure | AccessCardiology |...
It would be reasonable in a patient with multivessel CAD and a significant reduction in left ventricular (LV) function to presume that his/her cardiac dysfunction is directly related to myocardial ischemia resulting from a decrease in coronary blood flow and coronary flow reserve (CFR). Although all of the following are associated with LV dysfunction, whether the myocardium is stunned, hibernating, or infarcted depends on the duration, chronicity, severity, and repetitiveness of the myocardial ischemia (Figure 35-1). These different states of the myocardium can also coexist in different parts of the heart. ...
Hyperglycemia Has a Greater Impact on Left Ventricle Function in South Asians Than in Europeans [2014] | eCALDHyperglycemia Has a Greater Impact on Left Ventricle Function in South Asians Than in Europeans [2014] | eCALD
Chloe M. Park, Therese Tillin, Katherine March, Arjun K. Ghosh, Siana Jones, Andrew Wright, John Heasman, Darrel Francis, Naveed Sattar, Jamil Mayet, Nish Chaturvedi & Alun D. Hughes ...
Mechanisms underpinning exercise-induced changes in left ventricular function.Mechanisms underpinning exercise-induced changes in left ventricular function.
Despite a growing body of evidence suggesting that prolonged strenuous exercise (PSE) is associated with a transient reduction in right (RV) and left ventricular (LV) performance, the exact mechanism(s) responsible for this phenomenon is not fully un
US Patent # 8,150,513. Method and apparatus for assessing left ventricular function and optimizing cardiac pacing...US Patent # 8,150,513. Method and apparatus for assessing left ventricular function and optimizing cardiac pacing...
A system and method for monitoring left ventricular (LV) lateral wall motion and for optimizing cardiac pacing intervals based on left ventricular lateral wall motion is provided. The system includes
Left ventricular function and cardiac biomarker release - the influence of exercise intensity, duration and mode: a systematic...Left ventricular function and cardiac biomarker release - the influence of exercise intensity, duration and mode: a systematic...
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Vieillissement de la fonction cardiaque chez lhomme. | DIAL.pr - BOREALVieillissement de la fonction cardiaque chez l'homme. | DIAL.pr - BOREAL
In normal subjects at rest neither heart rate nor ejection volume are influenced by age. The loss of elasticity of the great arteries, and in particular the aorta which becomes tortuous and wider, results in an increase of impedance at ejection. At systole time pressure rises in the whole cardiovascular system, so that the left ventricle is subjected to an increase of parietal tension to which it adapts itself by hypertrophy which normalizes this tension. Ejection fraction and end-systolic volume are thus preserved, and the systolic function at rest globally remains unmodified by age. The delay and slowing down of relaxation due to hypertrophy of the left ventricle, to the reflection waves and to other changes in cardiac muscle physical properties during senescence reduce the importance of the initial phase of left ventricular filling. This major modification of diastolic dynamics at rest is compensated, at the end of diastole, by a more vigorous contraction of the left atrium, which increases ...
द्रव्यगुण शास्त्र - विकिपीडियाद्रव्यगुण शास्त्र - विकिपीडिया
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Lower Left Sided Abdominal Tender: 33 causesLower Left Sided Abdominal Tender: 33 causes
The freeMD virtual doctor has found 33 conditions that can cause Lower Left Sided Abdominal Tender. There are 5 common conditions that can cause Lower Left Sided Abdominal Tender. There are 3 somewhat common conditions that can cause Lower Left Sided Abdominal Tender. There are 10 uncommon conditions that can cause Lower Left Sided Abdominal Tender. There are 15 rare conditions that can cause Lower Left Sided Abdominal Tender.
TQUK Level 3 Award in Paediatric First Aid (RQF) - (PFA) Course Details | ProTrainings.euTQUK Level 3 Award in Paediatric First Aid (RQF) - (PFA) Course Details | ProTrainings.eu
Classroom course in tquk level 3 award in paediatric first aid (rqf) - (pfa) for anyone in the UK. Search for a course near you and get training by an approved ProTrainings instructor.
A new approach for evaluation of left ventricular diastolic function: Spatial and temporal analysis of left ventricular filling...A new approach for evaluation of left ventricular diastolic function: Spatial and temporal analysis of left ventricular filling...
TY - JOUR. T1 - A new approach for evaluation of left ventricular diastolic function. T2 - Spatial and temporal analysis of left ventricular filling flow propagation by color M-mode Doppler echocardiography. AU - Takatsuji, Hiroya. AU - Mikami, Taisei. AU - Urasawa, Kazushi. AU - Teranishi, Jun Ichi. AU - Onozuka, Hisao. AU - Takagi, Chika. AU - Makita, Yasuhiro. AU - Matsuo, Hisashi. AU - Kusuoka, Hideo. AU - Kitabatake, Akira. PY - 1996/2. Y1 - 1996/2. N2 - Objectives. To evaluate left ventricular diastolic function and differentiate the pseudonormalized transmitral flow pattern from the normal pattern, the propagation of left ventricular early filling flow was assessed quantitatively using color M-mode Doppler echocardiography. Background. Because the propagation of left ventricular early filling flow is disturbed in the left ventricle with impaired relaxation, quantification of such alterations should provide useful indexes for the evaluation of left ventricular diastolic function Methods. ...
Echocardiographic assessment of left ventricular function in coronary arterial disease. | HeartEchocardiographic assessment of left ventricular function in coronary arterial disease. | Heart
This investigation was designed to determine the role of echocardiography in the assessment of left ventricular function in patients with significant coronary arterial disease. Satisfactory echocardiograms were obtained in 43 patients with coronary arterial disease. The ventriculographic ejection fraction was determined by the area length method. The echocardiographic left ventricular end-diastolic dimension was increased to more than 5-4 cm in 17 patients. Fifteen of these patients had an ejection fraction of 0-45 or less. Three patients had a normal left ventricular end-diastolic dimension but an ejection fraction of less than 0-45. Twenty-three patients had an ejection fraction of more than 0-45 and a normal left ventricular end-diastolic dimension. The left ventricular end-diastolic dimension index was increased (greater than 3 cm/m2) in 15 patients, all of whom had ejection fraction of less than 0-45. Three patients had a normal left ventricular end-diastolic dimension index and an ejection ...
Left ventricular function at rest and during exercise in acute hypothyroidism. | HeartLeft ventricular function at rest and during exercise in acute hypothyroidism. | Heart
The effect of hypothyroidism on left ventricular function at rest and during exercise was studied in nine patients without demonstrable cardiovascular disease who had had total thyroidectomy and ablative radioiodine treatment for thyroid cancer. Radionuclide ventriculography and simultaneous right heart catheterisation were performed while the patients were hypothyroid two weeks after stopping triiodothyronine treatment (to permit routine screening for metastases) and while they were euthyroid on thyroxine replacement treatment. When the patients were hypothyroid, cardiac output, stroke volume, and end diastolic volume at rest were all lower and peripheral resistance was higher than when they were euthyroid. Pulmonary capillary wedge pressure, right atrial pressure, heart rate, left ventricular ejection fraction, and the systolic pressure:volume relation of the left ventricle, which was used as an estimate of the contractile state, were not significantly different when the patients were ...
Impact of Lifelong Exercise Training Dose on Ventricular-Arterial Coupling<...Impact of Lifelong Exercise Training Dose on Ventricular-Arterial Coupling<...
TY - JOUR. T1 - Impact of Lifelong Exercise Training Dose on Ventricular-Arterial Coupling. AU - Hieda, Michinari. AU - Howden, Erin. AU - Shibata, Shigeki. AU - Fujimoto, Naoki. AU - Bhella, Paul S.. AU - Hastings, Jeffrey L. AU - Tarumi Ph.D., Takashi. AU - Sarma, Satyam. AU - Fu, Qi. AU - Zhang, Rong. AU - Levine, Benjamin D. PY - 2018/12/4. Y1 - 2018/12/4. N2 - BACKGROUND: The dynamic Starling mechanism, as assessed by beat-by-beat changes in stroke volume and left ventricular end-diastolic pressure, reflects ventricular-arterial coupling. It deteriorates with age, and is preserved in highly trained masters athletes. Currently, it remains unclear how much exercise over a lifetime is necessary to preserve efficient ventricular-arterial coupling. The purpose of this study was to assess the dose-dependent relationship between lifelong exercise training and the dynamic Starling mechanism in healthy seniors. METHODS: One hundred two seniors were recruited and stratified into 4 groups based on 25 ...
Value of novel cardiac magnetic resonance indices for the diagnosis of acute myocarditis: Left ventricular mechanics and...Value of novel cardiac magnetic resonance indices for the diagnosis of acute myocarditis: Left ventricular mechanics and...
TY - JOUR. T1 - Value of novel cardiac magnetic resonance indices for the diagnosis of acute myocarditis: Left ventricular mechanics and parametric mapping imaging. AU - Nucifora, Gaetano. AU - Gilbert, Carl. AU - Selvanayagam, Joseph. PY - 2016. Y1 - 2016. U2 - 10.1016/j.ijcard.2016.08.039. DO - 10.1016/j.ijcard.2016.08.039. M3 - Article. VL - 223. SP - 881. EP - 882. JO - International Journal of Cardiology. JF - International Journal of Cardiology. SN - 0167-5273. ER - ...
PROGNOSTIC STRATIFICATION IN PATIENTS WITH ST-ELEVATION MYOCARDIAL INFACTION OVER TRANSTHORACIC ECHOCARDIOGRAPHY BY CMR | JACC:...PROGNOSTIC STRATIFICATION IN PATIENTS WITH ST-ELEVATION MYOCARDIAL INFACTION OVER TRANSTHORACIC ECHOCARDIOGRAPHY BY CMR | JACC:...
Background: We sought to evaluate the additional value of a multiparametric cardiac magnetic resonance (CMR) score in comparison with traditional TIMI (Thromolysis in Myocardial Infarction) score and transthoracic echocardiography (TTE) score in prognostic stratification of STEMI patients.. Methods: 209 STEMI patients reperfused by primary PCI underwent TTE and CMR 3 days after the index event. We measured: TIMI score, left ventricle ejection fraction (EFTTE), left ventricle end systolic volume (LVESVTTE) and number of myocardial segment with wall motion abnormalities (WMITTE) measured by TTE, left ventricle ejection fraction (EFCMR), left ventricle end systolic volume (LVESVCMR) and number of myocardial segment with wall motion abnormalities (WMICMR) measured by CMR, myocardial salvage index (MSI) and presence of microvascular obstruction (MVO). The primary clinical endpoint of study was the occurrence of major adverse cardiac events(MACE) defined as combined endpoint of hospitalization, acute ...
DIAFAX I+II-Effects of Inhalative Anesthetics on Left Ventricular Diastolic Function - Full Text View - ClinicalTrials.govDIAFAX I+II-Effects of Inhalative Anesthetics on Left Ventricular Diastolic Function - Full Text View - ClinicalTrials.gov
Anaesthetics may impair the diastolic function of the heart, but the importance of this finding for patients has not been sufficiently examined. Specially the effects on diastolic function in patients with diastolic dysfunction has to be determined. The aim of this study is to examine the effect of isoflurane (only part I), sevoflurane and desflurane (part I+II) on the diastolic left ventricular function by doppler echocardiography ...
eCite - Reliability of updated left ventricular diastolic function recommendations in predicting elevated left ventricular...eCite - Reliability of updated left ventricular diastolic function recommendations in predicting elevated left ventricular...
Reliability of updated left ventricular diastolic function recommendations in predicting elevated left ventricular filling pressure and prognosis
Pioglitazone improves left ventricular diastolic function in subjects with diabetes<...Pioglitazone improves left ventricular diastolic function in subjects with diabetes<...
TY - JOUR. T1 - Pioglitazone improves left ventricular diastolic function in subjects with diabetes. AU - Clarke, Geoffrey D. AU - Solis-Herrera, Carolina. AU - Molina-Wilkins, Marjorie. AU - Martinez, Sandra. AU - Merovci, Aurora. AU - Cersosimo, Eugenio. AU - Chilton, Robert J. AU - Iozzo, Patricia. AU - Gastaldelli, Amalia. AU - Abdul-ghani, Muhammad A. AU - Defronzo, Ralph A. PY - 2017/11/1. Y1 - 2017/11/1. N2 - OBJECTIVE To examine the effect of pioglitazone on myocardial insulin sensitivity and left ventricular (LV) function in patients with type 2 diabetes (T2D). RESEARCH DESIGN AND METHODS Twelve subjectswith T2Dand 12with normal glucose tolerance received a euglycemic insulin clamp. Myocardial glucose uptake (MGU) and myocardial perfusion were measured with [18F]fluoro-2-deoxy-D-glucose and [15O]H2O positron emission tomography before and after 24 weeks of pioglitazone treatment.Myocardial function and transmitral early diastolic relation/atrial contraction (E/A) flow ratio were ...
Electrocardiographic strain pattern and left ventricular diastolic function in hypertensive patients with left ventricular...Electrocardiographic strain pattern and left ventricular diastolic function in hypertensive patients with left ventricular...
Electrocardiographic strain pattern and left ventricular diastolic function in hypertensive patients with left ventricular hypertrophy: The LIFE study Academic Article ...
Lirias: Preoperative Systolic Strain Rate Predicts Postoperative Left Ventricular Systolic Function in Patients With Chronic...Lirias: Preoperative Systolic Strain Rate Predicts Postoperative Left Ventricular Systolic Function in Patients With Chronic...
Preoperative Systolic Strain Rate Predicts Postoperative Left Ventricular Systolic Function in Patients With Chronic Mitral Regurgitation: 18 Months Follow up ...
Machine learning analysis of left ventricular function to characterize heart failure with preserved ejection fractionMachine learning analysis of left ventricular function to characterize heart failure with preserved ejection fraction
Citation: Sanchez-Martinez S, Duchateau N, Erdei T, Kunszt G, Aakhus S, Degiovanni A, Marino P, Carluccio E, Piella G, Fraser AG, Bijnens BH. Machine learning analysis of left ventricular function to characterize heart failure with preserved ejection fraction. Circ Cardiovasc Imaging. 2018 Apr 16;11(4):e007138. DOI: 10.1161/CIRCIMAGING. ...
Left ventricular function changes after cardiomyoplasty in pat...Left ventricular function changes after cardiomyoplasty in pat...
Left ventricular function changes after cardiomyoplasty in patients with dilated cardiomyopathy.: Dynamic cardiomyoplasty has been reported in the treatment of
Quantification of left ventricular function and mass in cardiac Dual-Source CT (DSCT) exams: comparison of manual and...Quantification of left ventricular function and mass in cardiac Dual-Source CT (DSCT) exams: comparison of manual and...
All information about the latest scientific publications of the Clínica Universidad de Navarra. Quantification of left ventricular function and mass in cardiac Dual-Source CT (DSCT) exams: comparison of manual and semiautomatic segmentation algorithms
left ventricular function - Heart Disease - MedHelpleft ventricular function - Heart Disease - MedHelp
I have continuous pacing following AV node ablation two years ago and have recently developed symptoms suggesting perhaps reduced left ventricular function. I have read some literature which gives com...
Left atrioventricular remodeling in the assessment of the left ventricle diastolic function in patients with heart failure : a...Left atrioventricular remodeling in the assessment of the left ventricle diastolic function in patients with heart failure : a...
Multiparametric echocardiographic imaging of the failing heart is now increasingly used and useful in decision making in heart failure. The reasons for this, relies on the need of different strategies of handling these patients, as differentiation of systolic or diastolic dysfunction, as well as on the gamma of approaches available, such as percutaneous and surgical revascularization, devices implantations, and valvular regurgitations and stenosis corrections. Congestive heart failure in patients with normal left ventricular diameters or preserved left ventricular ejection fraction had been pointed out recently as present in a proportion so high as 40 to 50 percent of cases of heart failure, mainly due to the epidemics in well developed countries, as is the problem of not well controlled metabolic states (such as obesity and diabetes), but also due to the real word in developing countries, as is the case of hypertension epidemics and its lack of adequate control. As a matter of public utility, ...
Acute and Chronic Effect of His-pacing in Consecutive Patients With AV-block - Full Text View - ClinicalTrials.govAcute and Chronic Effect of His-pacing in Consecutive Patients With AV-block - Full Text View - ClinicalTrials.gov
Conventional right ventricular apical pacing may result in asynchronous ventricular contraction with delayed left ventricular activation, interventricular motion abnormalities, and worsening of left ventricular ejection fraction. His pacing is preserving a synchronous contraction and may prevent a decrease in left ventricular ejection fraction.. The aims of our crossover, double-blinded, randomized study is to evaluate the feasibility and long-term safety of permanent His pacing and to compare the effects on left ventricular ejection fraction of permanent His pacing with those of conventional right septal stimulation after 12 months treatment in patients with 2nd or 3rd degree AV-block. ...
Reduced Left Ventricular Functional Reserve in Hypertensive Patients With Preserved Function at Rest | HypertensionReduced Left Ventricular Functional Reserve in Hypertensive Patients With Preserved Function at Rest | Hypertension
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Left ventricular response to pressure afterload in children : aortic stenosis and coarctation A systematic review of the...Left ventricular response to pressure afterload in children : aortic stenosis and coarctation A systematic review of the...
Background:. Coarctation of the aorta (CoA) is a congenital heart disease which represents a narrowing of the proximal descending aorta, hence increasing pressure afterload to the left ventricle (LV). Conventional treatment of native CoA is surgical repair, however potential recurrence or other related complications e.g. aortic rupture, heart failure and cerebrovascular events are common. Thus, lifelong follow-up of these patients is required. Echocardiography is the most patients friendly method to evaluate CoA and in particular its effect on LV function. Moreover, the novel speckle tracking echocardiography (STE) is an important method to assess subclinical LV dysfunction, a technique that promises better evaluation of LV function in these patients.. The aims of this thesis were to review the literature on LV function in children with CoA using myocardial deformation imaging technologies, hence, to better understand the current knowledge and vagueness of the scientific evidence. We also aimed ...
Insulin resistance and glycemic abnormalities are associated with deterioration of left ventricular diastolic function: a cross...Insulin resistance and glycemic abnormalities are associated with deterioration of left ventricular diastolic function: a cross...
The main finding of the present study is that IR is associated with LVDD independent of overt diabetes. These finding persist after adjustment for CAD, hypertension, age, sex, history of previous myocardial infarction, history of previous coronary angioplasty, EF and glycaemic control. To our knowledge, this is the first study that demonstrated an association in a population of patients without a history of diabetes focusing on the published current guidelines for the diagnosis of LVDD.. Furthermore, our date confirm the observations that T2DM is associated with LVDD [9], which is considered a precursor of diabetic cardiomyopathy. In addition, we were able to extend the findings in previous studies showing the association between LVDD and IGT.. These findings are in line with a limited number of studies that assessed the relationship between prediabetes and LVDD mainly in population based studies [11]. Nevertheless, there are some concerns about methodological issues involving the identification ...
Left Ventricle Function, Definition & Anatomy | Body Maps - muslimselfportrait.infoLeft Ventricle Function, Definition & Anatomy | Body Maps - muslimselfportrait.info
The left ventricle is one of four chambers of the heart. It is located in the bottom left portion of the heart below the left atrium, separated by the mitral valve. As the heart contracts, blood eventually flows back into the left atrium, and then through the mitral valve, whereupon
Tests to evaluate left ventricular systolic functionTests to evaluate left ventricular systolic function
Assessment of left ventricular (LV) systolic function is important for diagnosis, management, follow-up, and prognostic evaluation of patients in a variety of clinical settings. Accurate and reliable determination of LV systolic function is important
Norepinephrine Induces Systolic Failure and Inhibits Antiapoptotic Gen by Mani Chopra, Padmalaya Das et al."Norepinephrine Induces Systolic Failure and Inhibits Antiapoptotic Gen" by Mani Chopra, Padmalaya Das et al.
AIMS: We examined the effect of norepinephrine (NE) infusion on left ventricular function and apoptotic genes during progression of polymicrobial sepsis. METHODS: Male Sprague-Dawley rats (350-400 g) were made septic by intraperitoneal (i.p.) administration of 200mg/kg cecal inoculum. Sham animals received 5% dextrose water, i.p. Echocardiography was performed at baseline, 3 days and 7 days post-sepsis/sham. NE (0.6 μgkg(-1)h(-1)) was infused for 2h, before the end of day 3 of echocardiography. At the end of day 7, rats were euthanized and heart tissues harvested for isolation of total RNA. PCR was performed using RT(2) profiler™ PCR array PARN-012 (Rat apoptosis array; SuperArray, MD) using RT(2) Real-Time™ SYBR Green PCR master mix PA-012. KEY FINDINGS: NE-infusion resulted in a significant decrease in the left ventricular ejection fraction (EF) (62.56±2.07 from the baseline 71.11±3.23, p SIGNIFICANCE: The data suggest that upregulation of a series of pro-apoptotic molecules could be
Third Ventricle Function, Anatomy & Definition | Body MapsThird Ventricle Function, Anatomy & Definition | Body Maps
The third ventricle is one of the four ventricles in the brain that communicate with one another. As with the other ventricles of the brain, it is filled with cerebrospinal fluid, which helps to protect the brain from injury and transport nutrients and waste.
Ejection fractionEjection fraction
The ejection fraction is a measurement of the hearts efficiency and can be used to estimate the function of the left ventricle, which pumps blood to the rest of the body.. The left ventricle pumps only a fraction of the blood it contains. The ejection fraction is the amount of blood pumped divided by the amount of blood the ventricle contains. A normal ejection fraction is more than 55% of the blood volume. If the heart becomes enlarged, even if the amount of blood being pumped by the left ventricle remains the same, the relative fraction of blood being ejected decreases. For example:. ...
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Physiological effects of percutaneous interventions / Coronary stenting in challenging cases 1244 , Loss of functional recovery In regional wall motion Is predictive for restenosis of the supplying angloplasty vessel - follow-up echocardtographlc study after high-risk angloplasty Adrian C. Borges, Franz Xaver Kleber, Dorothee SchOrger, Qert Baumann, Rosa Sicari, Alessandro Plngitore, Eugenio Rcano 1 . Medical Department I, Humbotdt-Unlverslty BerSn (Charite), Germany;1 Physiologic CSnic, CNR Pisa, Italy, on behalf of the VIDA (Viability Identification with Dobutantine-CHpyiidamote Adrrfnistmtion}Study Group The prediction of functional regional recovery Is possible wtth pharmacological stress echocarcbography and has In patients with severe left ventricular dysfunction and coronary artery disease a great prognostic Impact The aim of the study was to evaluate If the loss of recovery of regional left ventricular function after successful revascuiarization can predict a restenosis of the target ...
Assessment of LV Systolic Function - ppt video online downloadAssessment of LV Systolic Function - ppt video online download
Overview Normal LV contraction Global & regional indices of LV systolic Function Angiographic assessments Echocardiography MRI Computed Tomography Nuclear Imaging Take home message
Myocardial steatosis, impaired energetics and reduced circumferential strain are early manifestations of diabetic...Myocardial steatosis, impaired energetics and reduced circumferential strain are early manifestations of diabetic...
Diabetic patients were well-matched with controls (Figure 1). Myocardial energetics were disturbed (PCr/ATP ratio: 1.44 ± 0.35 vs. 1.98 ± 0.18, p , 0.001) and myocardial triglyceride content increased (1.06 ± 0.61 vs. 0.48 ± 0.24%, p = 0.01), despite the relatively short disease duration. Peak systolic circumferential strain was reduced, indicating subtle regional LV dysfunction. However, left ventricular volumes, mass and ejection fraction, as well as echocardiographic indices of diastolic function were similar in both groups. Furthermore, despite the metabolic abnormalities observed in diabetics, there was no difference in native T1 values (as a measure of myocardial fibrosis) between diabetic patients and controls (1181 ± 28 ms vs. 1195 ± 33 ms, p = 0.30 ...
Evaluation of two-dimensional strain rate imaging on left ventricular longaxis systolic function in patients with slow coronary...Evaluation of two-dimensional strain rate imaging on left ventricular longaxis systolic function in patients with slow coronary...
Objective: To evaluate the value of two-dimensional strain rate imaging (STE) in detecting the alteration of regionally left ventricular long-axis sys..
Alterations of LVEDP during IR; left ventricular end di | Open-iAlterations of LVEDP during IR; left ventricular end di | Open-i
Alterations of LVEDP during IR; left ventricular end diastolic pressure (LVEDP); control-ischemia reperfusion (C-IR), C-ischemic postconditioning (C-IPost), hyp
Minimisation: the platinum standard for trials? | The BMJMinimisation: the platinum standard for trials? | The BMJ
When we have to decide which of two drugs, interventions, or management strategies is the better, the most secure evidence is generally obtained from a randomised controlled trial. The primary objective of randomisation is to ensure that all other factors that might influence the outcome will be equally represented in the two groups, leaving the treatment under test as the only dissimilarity. Any difference in outcome can then be attributed to the treatment effect. But how realistic is this assumption in practice?. When published a randomised trial typically includes a table listing all the prior factors known actually or possibly to influence outcome. The average age and its distribution in each group and the proportion of men and women usually head the list, followed by other likely determinants of outcome. In the case of heart disease these will probably include details of left ventricular function; the proportions in each group with diabetes, hypertension, hyperlipidaemia, or a smoking ...
First Validated Risk Tool for Frequent PVCs Shows PotentialFirst Validated Risk Tool for Frequent PVCs Shows Potential
The ABC-VT score accurately predicts adverse events in patients with frequent PVCs and preserved left ventricular function -- a group whose management remains unclear.
गणितीय नियतांक - विकिपीडियागणितीय नियतांक - विकिपीडिया
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... - Answered by a verified Eye Doctor
They Left Out a StepThey Left Out a Step
That part where Mulder, with practically no information, and in the first five to ten minutes, comes up with the most ridiculous, implausible and unlikely explanation possible. Which, of course, turns out to be the right one. (Blastr; 10 steps that defined every X-Files episode) ...
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Is Time-Discounting Hyperbolic or Subadditive? | SpringerLinkIs Time-Discounting Hyperbolic or Subadditive? | SpringerLink
Subadditive time discounting means that discounting over a delay is greater when the delay is divided into subintervals than when it is left undivided. This may produce the most important result usual
Heart rateHeart rate
HIV associated cardiomyopathyHIV associated cardiomyopathy
K. A. AbrahamK. A. Abraham
Coarctation of the aortaCoarctation of the aorta
Steroid use in American footballSteroid use in American football
LapatinibLapatinib
Heart failure with preserved ejection fractionHeart failure with preserved ejection fraction
Peripartum cardiomyopathyPeripartum cardiomyopathy
TrimetazidineTrimetazidine
Cardiac amyloidosisCardiac amyloidosis
Anabolic steroidAnabolic steroid
Systolic heart murmurSystolic heart murmur
Howard ApfelHoward Apfel
Cardiac rhythm problems during space flightCardiac rhythm problems during space flight
Complications of hypertensionComplications of hypertension
ANKRD1ANKRD1
ANKRD2ANKRD2
Sándor J. KovácsSándor J. Kovács
End-systolic volumeEnd-systolic volume
CXL 1020CXL 1020
Pressure-volume loop experimentsPressure-volume loop experiments
Cardiac contractility modulationCardiac contractility modulation
Ejection fractionEjection fraction
Stroke volumeStroke volume
Cardiac outputCardiac output
End-diastolic volumeEnd-diastolic volume
Radionuclide angiographyRadionuclide angiography
Preload (cardiology)Preload (cardiology)
DiastoleDiastole
Fetal aortic stenosisFetal aortic stenosis
ReptileReptile
Artificial cardiac pacemakerArtificial cardiac pacemaker
Intravenous therapyIntravenous therapy
Epigenetics of neurodegenerative diseasesEpigenetics of neurodegenerative diseases
TachycardiaTachycardia
Common ostrichCommon ostrich
कार्दियाक अरिदमियाकार्दियाक अरिदमिया
Vitamin D toxicityVitamin D toxicity
Heart valveHeart valve
Hypertensive emergencyHypertensive emergency
ഫലകം:Cardiac procedures - വിക്കിപീഡിയഫലകം:Cardiac procedures - വിക്കിപീഡിയ
BirdBird
PericardiumPericardium
Atrial fibrillationAtrial fibrillation
Exercise-induced pulmonary hemorrhageExercise-induced pulmonary hemorrhage
Hypoxia (medical)Hypoxia (medical)
Single-photon emission computed tomographySingle-photon emission computed tomography
SepsisSepsis
Heart valveHeart valve
Idiopathic intracranial hypertensionIdiopathic intracranial hypertension
Mitral valveMitral valve
Birth defectBirth defect
Blood pressureBlood pressure
Ventricle (heart)Ventricle (heart)
Medical ultrasoundMedical ultrasound
ಹೃದಯಾಘಾತ - ವಿಕಿಪೀಡಿಯಹೃದಯಾಘಾತ - ವಿಕಿಪೀಡಿಯ
Stem-cell therapyStem-cell therapy
Radiofrequency ablationRadiofrequency ablation
GlyphosateGlyphosate
AnatomyOrganismsDiseasesChemicals and DrugsAnalytical, Diagnostic and Therapeutic Techniques and EquipmentPhenomena and ProcessesAnthropology, Education, Sociology and Social PhenomenaInformation ScienceHealth Care
Ventricular Function, LeftVentricular Function, RightVentricular Dysfunction, LeftVentricular FunctionStroke VolumeHeart VentriclesEchocardiographyHeartMyocardial ContractionMyocardial InfarctionVentricular Dysfunction, RightSystoleHemodynamicsDiastoleRadionuclide VentriculographyHeart FailureMyocardiumGated Blood-Pool ImagingElectrocardiographyRadionuclide AngiographyEchocardiography, DopplerCoronary CirculationHeart Function TestsHypertrophy, Left VentricularCardiomyopathy, DilatedCardiac CatheterizationVentricular RemodelingCineangiographyTime FactorsCoronary DiseaseHeart RateCardiac VolumeVentriculography, First-PassVentricular PressureCardiac OutputCardiomyopathiesEchocardiography, Doppler, PulsedMyocardial IschemiaTreatment OutcomeDobutamineFollow-Up StudiesAngiocardiographyExercise TestBlood PressureAtrial Function, LeftProspective StudiesCardiotonic AgentsVentricular DysfunctionCoronary AngiographyMagnetic Resonance Imaging, CineCoronary Artery BypassEchocardiography, Doppler, ColorCardiac Pacing, ArtificialTomography, Emission-Computed, Single-PhotonMitral Valve InsufficiencyPredictive Value of TestsPhonocardiographyMyocardial ReperfusionDogsMitral ValveGold RadioisotopesHeart DiseasesCoronary VesselsAortic Valve StenosisTricuspid ValveMyocardial Reperfusion InjuryAngioplasty, Balloon, CoronaryTechnetium Tc 99m SestamibiAngina PectorisAdrenergic beta-AntagonistsEchocardiography, StressPulmonary ArteryNatriuretic Peptide, BrainPrognosisRecovery of FunctionKinetocardiographyTetralogy of FallotCardiac Surgical ProceduresCineradiographyHeart AtriaTransposition of Great VesselsMyocardial StunningHeart AneurysmVentricular SeptumStreptokinaseBlood Flow VelocityArrhythmias, CardiacDisease Models, AnimalMyocardial RevascularizationTissue SurvivalPacemaker, ArtificialHeart SeptumHeart Valve Prosthesis ImplantationObserver VariationMetoprololEndocardiumCardiac Output, LowExercise ToleranceHeart Valve ProsthesisMyocarditisAnalysis of VarianceHeart Defects, CongenitalMyocytes, CardiacAortic ValveHypertrophy, Right VentricularChronic DiseaseAtrial FibrillationReproducibility of ResultsCoronary Artery DiseasePulmonary Valve InsufficiencySeverity of Illness IndexHypertension, PulmonaryThrombolytic TherapyElectrocardiography, AmbulatoryPostoperative PeriodRadiopharmaceuticalsPhysical ExertionPropanolaminesVascular ResistanceHeart Arrest, InducedCardiomegalyRetrospective StudiesThallium RadioisotopesHeart-Assist DevicesCardiomyopathy, HypertrophicAortic Valve InsufficiencyMagnetic Resonance ImagingOxygen ConsumptionHypoplastic Left Heart SyndromeFibrosisPostoperative ComplicationsCollateral CirculationTrimetazidineEchocardiography, TransesophagealPulmonary Wedge PressureCardiovascular AgentsEdema, CardiacTachycardia, VentricularFeasibility StudiesEchocardiography, Three-DimensionalAngiotensin-Converting Enzyme InhibitorsContrast MediaPericardiumFontan ProcedureRisk FactorsOrgan SizeRestHeart Valve DiseasesAnterior Wall Myocardial InfarctionCardiopulmonary BypassHeart BlockSurvival RateRandom AllocationSensitivity and SpecificityLinear ModelsCardiovascular Surgical ProceduresCoronary Vessel AnomaliesMyocardial Perfusion ImagingCarbazolesHeart TransplantationIodobenzenesImage Processing, Computer-AssistedTachycardiaHeart SoundsTomography, Emission-ComputedBundle-Branch BlockDouble-Blind MethodCase-Control StudiesTantalumOrganotechnetium CompoundsHypertensionDefibrillators, ImplantableBiological MarkersTomography, X-Ray ComputedPericardial EffusionCardioplegic SolutionsRats, WistarPressureAdrenergic beta-AgonistsMitral Valve StenosisAtrial Natriuretic FactorTricuspid Valve InsufficiencyEndocardial FibroelastosisAdrenergic beta-1 Receptor AgonistsRats, Sprague-DawleyPerfusionTakotsubo CardiomyopathyAnti-Arrhythmia AgentsVasodilator AgentsAcute DiseaseRisk AssessmentSwineIntra-Aortic Balloon PumpingCoronary StenosisStatistics, NonparametricModels, CardiovascularExtracorporeal CirculationScintillation CountingChi-Square DistributionAngiographyTechnetium
Mechanisms underpinning exercise-induced changes in left ventricular function.Mechanisms underpinning exercise-induced changes in left ventricular function.
US Patent # 8,150,513. Method and apparatus for assessing left ventricular function and optimizing cardiac pacing...US Patent # 8,150,513. Method and apparatus for assessing left ventricular function and optimizing cardiac pacing...
Ltered left ventricular function. This observation might be explained by the | IDH Inhibitor idhinhibitor.comLtered left ventricular function. This observation might be explained by the | IDH Inhibitor idhinhibitor.com
Role of Revascularization to Improve Left Ventricular Function | Color Atlas and Synopsis of Heart Failure | AccessCardiology |...Role of Revascularization to Improve Left Ventricular Function | Color Atlas and Synopsis of Heart Failure | AccessCardiology |...
Left ventricular function and cardiac biomarker release - the influence of exercise intensity, duration and mode: a systematic...Left ventricular function and cardiac biomarker release - the influence of exercise intensity, duration and mode: a systematic...
北京大学医学部机构知识库(IR@PKUHSC): INFLUENCE OF ABNORMAL FASTING PLASMA GLUCOSE ON LEFT VENTRICULAR FUNCTION IN OLDER PATIENTS WITH ACUTE...北京大学医学部机构知识库([email protected]): INFLUENCE OF ABNORMAL FASTING PLASMA GLUCOSE ON LEFT VENTRICULAR FUNCTION IN OLDER PATIENTS WITH ACUTE...
left ventricular function - Heart Disease - MedHelpleft ventricular function - Heart Disease - MedHelp
Tests to evaluate left ventricular systolic functionTests to evaluate left ventricular systolic function
Left Ventricular Form and Function | CirculationLeft Ventricular Form and Function | Circulation
Left ventricular systolic function after pulmonary valve replacement | EurekAlert! Science NewsLeft ventricular systolic function after pulmonary valve replacement | EurekAlert! Science News
Left ventricular function and exercise capacity.Left ventricular function and exercise capacity.
Echocardiographic Assessment of Left Ventricular Function in Type 1 Gauchers DiseaseEchocardiographic Assessment of Left Ventricular Function in Type 1 Gaucher's Disease
Pioglitazone Improves Left Ventricular Diastolic Function in Subjects With Diabetes | Diabetes CarePioglitazone Improves Left Ventricular Diastolic Function in Subjects With Diabetes | Diabetes Care
Echocardiographic Evaluation of Left Ventricular Diastolic Function | IntechOpenEchocardiographic Evaluation of Left Ventricular Diastolic Function | IntechOpen
Sex-dependent differences in left ventricular function and structure in chronic pressure overload. - PubMed - NCBISex-dependent differences in left ventricular function and structure in chronic pressure overload. - PubMed - NCBI
Frontiers | Left Ventricular Structure and Function in Elite Swimmers and Runners | PhysiologyFrontiers | Left Ventricular Structure and Function in Elite Swimmers and Runners | Physiology
Improvement in exercise performance by inhalation of methoxamine in patients with impaired left ventricular function. - PubMed...Improvement in exercise performance by inhalation of methoxamine in patients with impaired left ventricular function. - PubMed...
Left Ventricular Longitudinal Function Assessed by Speckle Tracking Ultrasound from a Single Apical Imaging PlaneLeft Ventricular Longitudinal Function Assessed by Speckle Tracking Ultrasound from a Single Apical Imaging Plane
Ultrasound Assessment of Left Ventricular Function Following Aortocoronary Saphenous Vein Bypass Grafting | CirculationUltrasound Assessment of Left Ventricular Function Following Aortocoronary Saphenous Vein Bypass Grafting | Circulation
Echocardiographic assessment of left ventricular function in coronary arterial disease. | HeartEchocardiographic assessment of left ventricular function in coronary arterial disease. | Heart
NIOSHTIC-2 Publications Search - 20039912 - Pulmonary function and left ventricular mass in African Americans: The...NIOSHTIC-2 Publications Search - 20039912 - Pulmonary function and left ventricular mass in African Americans: The...
Assessing left ventricular systolic function in shock : evaluation of echocardiographic parameters in intensive careAssessing left ventricular systolic function in shock : evaluation of echocardiographic parameters in intensive care
Reduced Left Ventricular Functional Reserve in Hypertensive Patients With Preserved Function at Rest | HypertensionReduced Left Ventricular Functional Reserve in Hypertensive Patients With Preserved Function at Rest | Hypertension
Effects of Sedentary Aging and Lifelong Exercise on Left Ventricular Systolic Function.Effects of Sedentary Aging and Lifelong Exercise on Left Ventricular Systolic Function.
Left ventricular function at rest and during exercise in acute hypothyroidism. | HeartLeft ventricular function at rest and during exercise in acute hypothyroidism. | Heart
Evaluation of left ventricular diastolic function: state of the art after 35 years with Doppler assessment | SpringerLinkEvaluation of left ventricular diastolic function: state of the art after 35 years with Doppler assessment | SpringerLink
DIAFAX I+II-Effects of Inhalative Anesthetics on Left Ventricular Diastolic Function - Full Text View - ClinicalTrials.govDIAFAX I+II-Effects of Inhalative Anesthetics on Left Ventricular Diastolic Function - Full Text View - ClinicalTrials.gov
93 Left ventricular function changes in a general pacemaker population93 Left ventricular function changes in a general pacemaker population
Left ventricular systolic function after pulmonary valve replacementLeft ventricular systolic function after pulmonary valve replacement
Aldosterone Blockade Ups Left Ventricular Diastolic Function - MPRAldosterone Blockade Ups Left Ventricular Diastolic Function - MPR
Re: Angiotensin receptor blocker in patients with ST segment elevation myocardial infarction with preserved left ventricular...Re: Angiotensin receptor blocker in patients with ST segment elevation myocardial infarction with preserved left ventricular...
Do patients with suspected heart failure and preserved left ventricular systolic function suffer from diastolic heart failure...Do patients with suspected heart failure and preserved left ventricular systolic function suffer from "diastolic heart failure"...
Evaluation of global left ventricular myocardial function with electrocardiogram-gated multidetector computed tomography:...Evaluation of global left ventricular myocardial function with electrocardiogram-gated multidetector computed tomography:...
Left ventricular function, mass, and volumes in type 1 diabetic patients - and relation to NT-proBNP | Diabetes CareLeft ventricular function, mass, and volumes in type 1 diabetic patients - and relation to NT-proBNP | Diabetes Care
An Overview of the Consequences of Distal Coronary Microembolization on Left Ventricular Function, Perfusion and ViabilityAn Overview of the Consequences of Distal Coronary Microembolization on Left Ventricular Function, Perfusion and Viability
AnatomyOrganismsDiseasesChemicals and DrugsAnalytical, Diagnostic and Therapeutic Techniques and EquipmentPhenomena and ProcessesAnthropology, Education, Sociology and Social PhenomenaInformation ScienceHealth Care
Ventricular Function, LeftVentricular Function, RightVentricular Dysfunction, LeftVentricular FunctionStroke VolumeHeart VentriclesEchocardiographyHeartMyocardial ContractionMyocardial InfarctionVentricular Dysfunction, RightSystoleHemodynamicsDiastoleRadionuclide VentriculographyHeart FailureMyocardiumGated Blood-Pool ImagingElectrocardiographyRadionuclide AngiographyEchocardiography, DopplerCoronary CirculationHeart Function TestsHypertrophy, Left VentricularCardiomyopathy, DilatedCardiac CatheterizationVentricular RemodelingCineangiographyTime FactorsCoronary DiseaseHeart RateCardiac VolumeVentriculography, First-PassVentricular PressureCardiac OutputCardiomyopathiesEchocardiography, Doppler, PulsedMyocardial IschemiaTreatment OutcomeDobutamineFollow-Up StudiesAngiocardiographyExercise TestBlood PressureAtrial Function, LeftProspective StudiesCardiotonic AgentsVentricular DysfunctionCoronary AngiographyMagnetic Resonance Imaging, CineCoronary Artery BypassEchocardiography, Doppler, ColorCardiac Pacing, ArtificialTomography, Emission-Computed, Single-PhotonMitral Valve InsufficiencyPredictive Value of TestsPhonocardiographyMyocardial ReperfusionDogsMitral ValveGold RadioisotopesHeart DiseasesCoronary VesselsAortic Valve StenosisTricuspid ValveMyocardial Reperfusion InjuryAngioplasty, Balloon, CoronaryTechnetium Tc 99m SestamibiAngina PectorisAdrenergic beta-AntagonistsEchocardiography, StressPulmonary ArteryNatriuretic Peptide, BrainPrognosisRecovery of FunctionKinetocardiographyTetralogy of FallotCardiac Surgical ProceduresCineradiographyHeart AtriaTransposition of Great VesselsMyocardial StunningHeart AneurysmVentricular SeptumStreptokinaseBlood Flow VelocityArrhythmias, CardiacDisease Models, AnimalMyocardial RevascularizationTissue SurvivalPacemaker, ArtificialHeart SeptumHeart Valve Prosthesis ImplantationObserver VariationMetoprololEndocardiumCardiac Output, LowExercise ToleranceHeart Valve ProsthesisMyocarditisAnalysis of VarianceHeart Defects, CongenitalMyocytes, CardiacAortic ValveHypertrophy, Right VentricularChronic DiseaseAtrial FibrillationReproducibility of ResultsCoronary Artery DiseasePulmonary Valve InsufficiencySeverity of Illness IndexHypertension, PulmonaryThrombolytic TherapyElectrocardiography, AmbulatoryPostoperative PeriodRadiopharmaceuticalsPhysical ExertionPropanolaminesVascular ResistanceHeart Arrest, InducedCardiomegalyRetrospective StudiesThallium RadioisotopesHeart-Assist DevicesCardiomyopathy, HypertrophicAortic Valve InsufficiencyMagnetic Resonance ImagingOxygen ConsumptionHypoplastic Left Heart SyndromeFibrosisPostoperative ComplicationsCollateral CirculationTrimetazidineEchocardiography, TransesophagealPulmonary Wedge PressureCardiovascular AgentsEdema, CardiacTachycardia, VentricularFeasibility StudiesEchocardiography, Three-DimensionalAngiotensin-Converting Enzyme InhibitorsContrast MediaPericardiumFontan ProcedureRisk FactorsOrgan SizeRestHeart Valve DiseasesAnterior Wall Myocardial InfarctionCardiopulmonary BypassHeart BlockSurvival RateRandom AllocationSensitivity and SpecificityLinear ModelsCardiovascular Surgical ProceduresCoronary Vessel AnomaliesMyocardial Perfusion ImagingCarbazolesHeart TransplantationIodobenzenesImage Processing, Computer-AssistedTachycardiaHeart SoundsTomography, Emission-ComputedBundle-Branch BlockDouble-Blind MethodCase-Control StudiesTantalumOrganotechnetium CompoundsHypertensionDefibrillators, ImplantableBiological MarkersTomography, X-Ray ComputedPericardial EffusionCardioplegic SolutionsRats, WistarPressureAdrenergic beta-AgonistsMitral Valve StenosisAtrial Natriuretic FactorTricuspid Valve InsufficiencyEndocardial FibroelastosisAdrenergic beta-1 Receptor AgonistsRats, Sprague-DawleyPerfusionTakotsubo CardiomyopathyAnti-Arrhythmia AgentsVasodilator AgentsAcute DiseaseRisk AssessmentSwineIntra-Aortic Balloon PumpingCoronary StenosisStatistics, NonparametricModels, CardiovascularExtracorporeal CirculationScintillation CountingChi-Square DistributionAngiographyTechnetium
DiastolicAtrialVentricleImprove left ventricular functionDysfunctionIschemiaMyocardial tissuePatientsWallMortalityTroponinEstimating left ventricular systolicDysfunctionHYPERTROPHYPreserved left ventricular systolic functionLVEFMyocardial infarctionCardiac functionAbstractLower left ventricularLongitudinalArterialParametersCONGESTIVE HEART FDiastolic left ventricular functionPostoperative left ventricular functionChanges in left ventricularHeart failureMyocardiumImpairmentGlobal left ventricularPhysiologyEchocardiographic left ventricularCardiovascularVolumesSTEMIImprovement in ventricularQuantificationDilationClinicalDilatationEvaluationSeptalVelocitiesAbnormalSeverelySignificantlyPosterior wall
Diastolic3
Atrial1
  • An automatic pacing interval optimization method evaluates the LV lateral wall motion during varying pacing interval settings, including atrial-ventricular intervals and inter-ventricular intervals and selects the pacing interval setting(s) that correspond to LV lateral wall motion associated with improved cardiac synchrony and hemodynamic performance. (patents.com)
Ventricle1
  • The system includes an implantable or external cardiac stimulation device in association with a set of leads including a left ventricular epicardial or coronary sinus lead equipped with a motion sensor electromechanically coupled to the lateral wall of the left ventricle. (patents.com)
Improve left ventricular function1
Dysfunction2
Ischemia1
Myocardial tissue1
Patients1
  • In patients with significant CAD and impaired LV function, considerations should be made as to whether they would benefit from coronary revascularization ( Figure 35-2 ). (mhmedical.com)
Wall1
Mortality1
Troponin1
Estimating left ventricular systolic1
Dysfunction32
HYPERTROPHY11
Preserved left ventricular systolic function8
LVEF6
Myocardial infarction13
Cardiac function9
Abstract7
Lower left ventricular1
Longitudinal8
Arterial6
Parameters8
CONGESTIVE HEART F2
Diastolic left ventricular function2
Postoperative left ventricular function1
Changes in left ventricular5
Heart failure11
Myocardium8
Impairment6
Global left ventricular2
  • Speckle Tracking Ultrasound (STU) is a novel method allowing assessment of both regional and global left ventricular function [ 18 - 21 ] in dedicated semiautomatic software, thereby making this method fast and potentially available for online real-time analysis in the critical setting where time is crucial. (hindawi.com)
  • Potential utility of global left ventricular end-systolic wall stress measured by CMR. (ebscohost.com)
Physiology1
Echocardiographic left ventricular2
Cardiovascular7
Volumes9
STEMI1
Improvement in ventricular2
Quantification4
Dilation2
Clinical12
Dilatation2
Evaluation5
Septal4
Velocities1
  • 51.7% women), we measured left atrial volume index (LAVI), peak velocities of the transmitral blood flow (E) and mitral annular movement (e0) in early diastole and E/e0 9.6 years (median) after CBP and ABP. (eur.nl)
Abnormal3
Severely3
Significantly5
Posterior wall2
Osteoprotegerin levels in ST-elevation myocardial infarction: Temporal profile and association with myocardial injury and left...
Osteoprotegerin levels in ST-elevation myocardial infarction: Temporal profile and association with myocardial injury and left... (journals.plos.org)
1991 - Volume 18 - Issue : Journal of Cardiovascular Pharmacology
1991 - Volume 18 - Issue : Journal of Cardiovascular Pharmacology (journals.lww.com)
Pioglitazone Improves Left Ventricular Diastolic Function in Subjects With Diabetes | Diabetes Care
Pioglitazone Improves Left Ventricular Diastolic Function in Subjects With Diabetes | Diabetes Care (care.diabetesjournals.org)
Long-term impact of multivessel disease on cause-specific mortality after ST elevation myocardial infarction treated with...
Long-term impact of multivessel disease on cause-specific mortality after ST elevation myocardial infarction treated with... (heart.bmj.com)
Plus it
Plus it (jpet.aspetjournals.org)
No Effect of Transcendental Meditation on Left Ventricular Function | Annals of Internal Medicine | American College of...
No Effect of Transcendental Meditation on Left Ventricular Function | Annals of Internal Medicine | American College of... (annals.org)
February 2014 - Volume 46 - Issue 2 : Medicine & Science in Sports & Exercise
February 2014 - Volume 46 - Issue 2 : Medicine & Science in Sports & Exercise (journals.lww.com)
Frontiers | Left Ventricular Structure and Function in Elite Swimmers and Runners | Physiology
Frontiers | Left Ventricular Structure and Function in Elite Swimmers and Runners | Physiology (frontiersin.org)
August 1995 - Volume 6 - Issue 8 : Coronary Artery Disease
August 1995 - Volume 6 - Issue 8 : Coronary Artery Disease (journals.lww.com)
Feasibility of a novel, synthetic, self-assembling peptide for suture-line haemostasis in cardiac surgery | SpringerLink
Feasibility of a novel, synthetic, self-assembling peptide for suture-line haemostasis in cardiac surgery | SpringerLink (link.springer.com)
Physical activity guidelines and cardiovascular risk in children: a cross sectional analysis to determine whether 60 minutes is...
Physical activity guidelines and cardiovascular risk in children: a cross sectional analysis to determine whether 60 minutes is... (bmcpublichealth.biomedcentral.com)
MacArthur SES & Health Network | Research
MacArthur SES & Health Network | Research (macses.ucsf.edu)
Echocardiographic Assessment of Left Ventricular Remodeling: Are Left Ventricular Diameters Suitable Tools? | JACC: Journal of...
Echocardiographic Assessment of Left Ventricular Remodeling: Are Left Ventricular Diameters Suitable Tools? | JACC: Journal of... (onlinejacc.org)
Echocardiography: future developments. What is diastole and how to assess it? Impaired left ventricular systolic function -...
Echocardiography: future developments. What is diastole and how to assess it? Impaired left ventricular systolic function -... (minervamedica.it)
CiteSeerX - Citation Query Finkbeiner WE et al. Effects of long-term right ventricular apical pacing on left ventricular...
CiteSeerX - Citation Query Finkbeiner WE et al. Effects of long-term right ventricular apical pacing on left ventricular... (citeseer.ist.psu.edu)
Randomized Clinical Trial of Intravenous Infusion Umbilical Cord Mesenchymal Stem Cells on Cardiopathy - Full Text View -...
Randomized Clinical Trial of Intravenous Infusion Umbilical Cord Mesenchymal Stem Cells on Cardiopathy - Full Text View -... (clinicaltrials.gov)
Coronary Artery Atherosclerosis: Practice Essentials, Background, Anatomy
Coronary Artery Atherosclerosis: Practice Essentials, Background, Anatomy (emedicine.medscape.com)
Left ventricular function - Heart Disease - MedHelp
Left ventricular function - Heart Disease - MedHelp (medhelp.org)
Plus it
Plus it (cmaj.ca)
Hypertension - Research database - University of Groningen
Hypertension - Research database - University of Groningen (rug.nl)
CV exercise betters cardiac aging in sedentary middle-aged adults
CV exercise betters cardiac aging in sedentary middle-aged adults (medicalxpress.com)
SNMMI and ASNC issue joint guidelines for quantification of myocardial blood flow using PET
SNMMI and ASNC issue joint guidelines for quantification of myocardial blood flow using PET (medicalxpress.com)