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TachycardiaTachycardia, VentricularTachycardia, SupraventricularEncyclopedias as TopicHeart RateTachycardia, Atrioventricular Nodal ReentryTachycardia, Ectopic AtrialDictionaries, MedicalDictionaries as TopicElectronic MailFood Dispensers, AutomaticEditorial PoliciesAuthorshipPeriodicals as TopicPostal ServiceInternetPostural Orthostatic Tachycardia SyndromeDizzinessTilt-Table TestHypotension, OrthostaticPostureSyndromeTachycardia, ParoxysmalWolff-Parkinson-White SyndromeCatheter AblationAtrioventricular NodeElectrocardiographyTachycardia, SinusFlecainideAnti-Arrhythmia AgentsChronobiology DisordersArrhythmias, CardiacElectrocoagulationAtrial FibrillationAblation TechniquesStellate GanglionCardiac Imaging TechniquesAtrial FlutterHeart Conduction SystemElectrocardiography, AmbulatoryAtrial Premature ComplexesCopyrightPatient Education as TopicUrology Department, HospitalHeartCaffeineCoffeeCacaoCarbonated BeveragesTea
Tachycardia: Abnormally rapid heartbeat, usually with a HEART RATE above 100 beats per minute for adults. Tachycardia accompanied by disturbance in the cardiac depolarization (cardiac arrhythmia) is called tachyarrhythmia.Tachycardia, Ventricular: An abnormally rapid ventricular rhythm usually in excess of 150 beats per minute. It is generated within the ventricle below the BUNDLE OF HIS, either as autonomic impulse formation or reentrant impulse conduction. Depending on the etiology, onset of ventricular tachycardia can be paroxysmal (sudden) or nonparoxysmal, its wide QRS complexes can be uniform or polymorphic, and the ventricular beating may be independent of the atrial beating (AV dissociation).Tachycardia, Supraventricular: A generic expression for any tachycardia that originates above the BUNDLE OF HIS.Encyclopedias as Topic: Works containing information articles on subjects in every field of knowledge, usually arranged in alphabetical order, or a similar work limited to a special field or subject. (From The ALA Glossary of Library and Information Science, 1983)Heart Rate: The number of times the HEART VENTRICLES contract per unit of time, usually per minute.Tachycardia, Atrioventricular Nodal Reentry: Abnormally rapid heartbeats caused by reentry of atrial impulse into the dual (fast and slow) pathways of ATRIOVENTRICULAR NODE. The common type involves a blocked atrial impulse in the slow pathway which reenters the fast pathway in a retrograde direction and simultaneously conducts to the atria and the ventricles leading to rapid HEART RATE of 150-250 beats per minute.Tachycardia, Ectopic Atrial: Abnormally rapid heartbeats originating from one or more automatic foci (nonsinus pacemakers) in the HEART ATRIUM but away from the SINOATRIAL NODE. Unlike the reentry mechanism, automatic tachycardia speeds up and slows down gradually. The episode is characterized by a HEART RATE between 135 to less than 200 beats per minute and lasting 30 seconds or longer.Dictionaries, MedicalDictionaries as Topic: Lists of words, usually in alphabetical order, giving information about form, pronunciation, etymology, grammar, and meaning.Electronic Mail: Messages between computer users via COMPUTER COMMUNICATION NETWORKS. This feature duplicates most of the features of paper mail, such as forwarding, multiple copies, and attachments of images and other file types, but with a speed advantage. The term also refers to an individual message sent in this way.Food Dispensers, Automatic: Mechanical food dispensing machines.Editorial Policies: The guidelines and policy statements set forth by the editor(s) or editorial board of a publication.Authorship: The profession of writing. Also the identity of the writer as the creator of a literary production.Periodicals as Topic: A publication issued at stated, more or less regular, intervals.Postal Service: The functions and activities carried out by the U.S. Postal Service, foreign postal services, and private postal services such as Federal Express.Internet: A loose confederation of computer communication networks around the world. The networks that make up the Internet are connected through several backbone networks. The Internet grew out of the US Government ARPAnet project and was designed to facilitate information exchange.Postural Orthostatic Tachycardia Syndrome: A syndrome of ORTHOSTATIC INTOLERANCE combined with excessive upright TACHYCARDIA, and usually without associated ORTHOSTATIC HYPOTENSION. All variants have in common an excessively reduced venous return to the heart (central HYPOVOLEMIA) while upright.Dizziness: An imprecise term which may refer to a sense of spatial disorientation, motion of the environment, or lightheadedness.Tilt-Table Test: A standard and widely accepted diagnostic test used to identify patients who have a vasodepressive and/or cardioinhibitory response as a cause of syncope. (From Braunwald, Heart Disease, 7th ed)Hypotension, Orthostatic: A significant drop in BLOOD PRESSURE after assuming a standing position. Orthostatic hypotension is a finding, and defined as a 20-mm Hg decrease in systolic pressure or a 10-mm Hg decrease in diastolic pressure 3 minutes after the person has risen from supine to standing. Symptoms generally include DIZZINESS, blurred vision, and SYNCOPE.Posture: The position or attitude of the body.Syndrome: A characteristic symptom complex.Tachycardia, Paroxysmal: Abnormally rapid heartbeats with sudden onset and cessation.Wolff-Parkinson-White Syndrome: A form of ventricular pre-excitation characterized by a short PR interval and a long QRS interval with a delta wave. In this syndrome, atrial impulses are abnormally conducted to the HEART VENTRICLES via an ACCESSORY CONDUCTING PATHWAY that is located between the wall of the right or left atria and the ventricles, also known as a BUNDLE OF KENT. The inherited form can be caused by mutation of PRKAG2 gene encoding a gamma-2 regulatory subunit of AMP-activated protein kinase.Catheter Ablation: Removal of tissue with electrical current delivered via electrodes positioned at the distal end of a catheter. Energy sources are commonly direct current (DC-shock) or alternating current at radiofrequencies (usually 750 kHz). The technique is used most often to ablate the AV junction and/or accessory pathways in order to interrupt AV conduction and produce AV block in the treatment of various tachyarrhythmias.Atrioventricular Node: A small nodular mass of specialized muscle fibers located in the interatrial septum near the opening of the coronary sinus. It gives rise to the atrioventricular bundle of the conduction system of the heart.Electrocardiography: Recording of the moment-to-moment electromotive forces of the HEART as projected onto various sites on the body's surface, delineated as a scalar function of time. The recording is monitored by a tracing on slow moving chart paper or by observing it on a cardioscope, which is a CATHODE RAY TUBE DISPLAY.Tachycardia, Sinus: Simple rapid heartbeats caused by rapid discharge of impulses from the SINOATRIAL NODE, usually between 100 and 180 beats/min in adults. It is characterized by a gradual onset and termination. Sinus tachycardia is common in infants, young children, and adults during strenuous physical activities.Flecainide: A potent anti-arrhythmia agent, effective in a wide range of ventricular and atrial ARRHYTHMIAS and TACHYCARDIAS.Anti-Arrhythmia Agents: Agents used for the treatment or prevention of cardiac arrhythmias. They may affect the polarization-repolarization phase of the action potential, its excitability or refractoriness, or impulse conduction or membrane responsiveness within cardiac fibers. Anti-arrhythmia agents are often classed into four main groups according to their mechanism of action: sodium channel blockade, beta-adrenergic blockade, repolarization prolongation, or calcium channel blockade.Chronobiology Disorders: Disruptions of the rhythmic cycle of bodily functions or activities.Arrhythmias, Cardiac: Any disturbances of the normal rhythmic beating of the heart or MYOCARDIAL CONTRACTION. Cardiac arrhythmias can be classified by the abnormalities in HEART RATE, disorders of electrical impulse generation, or impulse conduction.Electrocoagulation: Procedures using an electrically heated wire or scalpel to treat hemorrhage (e.g., bleeding ulcers) and to ablate tumors, mucosal lesions, and refractory arrhythmias. It is different from ELECTROSURGERY which is used more for cutting tissue than destroying and in which the patient is part of the electric circuit.Atrial Fibrillation: Abnormal cardiac rhythm that is characterized by rapid, uncoordinated firing of electrical impulses in the upper chambers of the heart (HEART ATRIA). In such case, blood cannot be effectively pumped into the lower chambers of the heart (HEART VENTRICLES). It is caused by abnormal impulse generation.Ablation Techniques: Removal of tissue by vaporization, abrasion, or destruction. Methods used include heating tissue by hot liquids or microwave thermal heating, freezing (CRYOABLATION), chemical ablation, and photoablation with LASERS.Stellate Ganglion: A paravertebral sympathetic ganglion formed by the fusion of the inferior cervical and first thoracic ganglia.Cardiac Imaging Techniques: Visualization of the heart structure and cardiac blood flow for diagnostic evaluation or to guide cardiac procedures via techniques including ENDOSCOPY (cardiac endoscopy, sometimes refered to as cardioscopy), RADIONUCLIDE IMAGING; MAGNETIC RESONANCE IMAGING; TOMOGRAPHY; or ULTRASONOGRAPHY.Atrial Flutter: Rapid, irregular atrial contractions caused by a block of electrical impulse conduction in the right atrium and a reentrant wave front traveling up the inter-atrial septum and down the right atrial free wall or vice versa. Unlike ATRIAL FIBRILLATION which is caused by abnormal impulse generation, typical atrial flutter is caused by abnormal impulse conduction. As in atrial fibrillation, patients with atrial flutter cannot effectively pump blood into the lower chambers of the heart (HEART VENTRICLES).Heart Conduction System: An impulse-conducting system composed of modified cardiac muscle, having the power of spontaneous rhythmicity and conduction more highly developed than the rest of the heart.Electrocardiography, Ambulatory: Method in which prolonged electrocardiographic recordings are made on a portable tape recorder (Holter-type system) or solid-state device ("real-time" system), while the patient undergoes normal daily activities. It is useful in the diagnosis and management of intermittent cardiac arrhythmias and transient myocardial ischemia.Atrial Premature Complexes: A type of cardiac arrhythmia with premature atrial contractions or beats caused by signals originating from ectopic atrial sites. The ectopic signals may or may not conduct to the HEART VENTRICLES. Atrial premature complexes are characterized by premature P waves on ECG which are different in configuration from the P waves generated by the normal pacemaker complex in the SINOATRIAL NODE.Copyright: It is a form of protection provided by law. In the United States this protection is granted to authors of original works of authorship, including literary, dramatic, musical, artistic, and certain other intellectual works. This protection is available to both published and unpublished works. (from Circular of the United States Copyright Office, 6/30/2008)Patient Education as Topic: The teaching or training of patients concerning their own health needs.Urology Department, Hospital: Hospital department responsible for the administration and provision of diagnostic and therapeutic services for the urologic patient.Heart: The hollow, muscular organ that maintains the circulation of the blood.Caffeine: A methylxanthine naturally occurring in some beverages and also used as a pharmacological agent. Caffeine's most notable pharmacological effect is as a central nervous system stimulant, increasing alertness and producing agitation. It also relaxes SMOOTH MUSCLE, stimulates CARDIAC MUSCLE, stimulates DIURESIS, and appears to be useful in the treatment of some types of headache. Several cellular actions of caffeine have been observed, but it is not entirely clear how each contributes to its pharmacological profile. Among the most important are inhibition of cyclic nucleotide PHOSPHODIESTERASES, antagonism of ADENOSINE RECEPTORS, and modulation of intracellular calcium handling.Coffee: A beverage made from ground COFFEA beans (SEEDS) infused in hot water. It generally contains CAFFEINE and THEOPHYLLINE unless it is decaffeinated.Cacao: A tree of the family Sterculiaceae (or Byttneriaceae), usually Theobroma cacao, or its seeds, which after fermentation and roasting, yield cocoa and chocolate.Carbonated Beverages: Drinkable liquids combined with or impregnated with carbon dioxide.Tea: The infusion of leaves of CAMELLIA SINENSIS (formerly Thea sinensis) as a beverage, the familiar Asian tea, which contains CATECHIN (especially epigallocatechin gallate) and CAFFEINE.

Site of myocardial infarction. A determinant of the cardiovascular changes induced in the cat by coronary occlusion. (1/1171)

The influence of site of acute myocardial infarction on heart rate, blood pressure, cardiac output, total peripheral resistance (TPR), cardiac rhythm, and mortality was determined in 58 anesthetized cats by occlusion of either the left anterior descending (LAD), left circumflex or right coronary artery. LAD occlusion resulted in immediate decrease in cardiac output, heart rate, and blood pressure, an increase in TPR, and cardiac rhythm changes including premature ventricular beats, ventricular tachycardia, and occasionally ventricular fibrillation. The decrease in cardiac output and increase in TPR persisted in the cats surviving a ventricular arrhythmia. In contrast, right coronary occlusion resulted in a considerably smaller decrease in cardiac output. TPR did not increase, atrioventricular condition disturbances were common, and sinus bradycardia and hypotension persisted in the cats recovering from an arrhythmia. Left circumflex ligation resulted in cardiovascular changes intermediate between those produced by occlusion of the LAD or the right coronary artery. Mortality was similar in each of the three groups. We studied the coronary artery anatomy in 12 cats. In 10, the blood supply to the sinus node was from the right coronary artery and in 2, from the left circumflex coronary artery. The atrioventricular node artery arose from the right in 9 cats, and from the left circumflex in 3. The right coronary artery was dominant in 9 cats and the left in 3. In conclusion, the site of experimental coronary occlusion in cats is a major determinant of the hemodynamic and cardiac rhythm changes occurring after acute myocardial infarction. The cardiovascular responses evoked by ligation are related in part to the anatomical distribution of the occluded artery.  (+info)

Regional differences in the recovery course of tachycardia-induced changes of atrial electrophysiological properties. (2/1171)

BACKGROUND: Regional differences in recovery of tachycardia-induced changes of atrial electrophysiological properties have not been well studied. METHODS AND RESULTS: In the control group (5 dogs), atrial effective refractory period (AERP) and inducibility of atrial fibrillation (AF) were assessed before and every 4 hours for 48 hours after complete atrioventricular junction (AVJ) ablation with 8-week VVI pacing. In experimental group 1 (15 dogs), AERP and inducibility of AF were assessed before and after complete AVJ ablation with 8-week rapid right atrial (RA) pacing (780 bpm) and VVI pacing. In experimental group 2 (7 dogs), AERP and inducibility of AF were assessed before and after 8-week rapid left atrial (LA) pacing and VVI pacing. AERP and inducibility and duration of AF were obtained from 7 epicardial sites. In the control group, atrial electrophysiological properties obtained immediately and during 48-hour measurements after pacing did not show any change. In the 2 experimental groups, recovery of atrial electrophysiological properties included a progressive recovery of AERP shortening, recovery of AERP maladaptation, and decrease of duration and episodes of reinduced AF. However, recovery of shortening and maladaptation of AERP and inducibility of AF was slower at the LA than at the RA and Bachmann's bundle. CONCLUSIONS: The LA had a slower recovery of tachycardia-induced changes of atrial electrophysiological properties, and this might play a critical role in initiation of AF.  (+info)

Ketotifen and cardiovascular effects of xamoterol following single and chronic dosing in healthy volunteers. (3/1171)

AIMS: To study whether desensitization occurs after long-term administration of the 1-adrenoceptor partial agonist xamoterol and, if so, whether this can be influenced by ketotifen. METHODS: In a double-blind, randomized design 10 young, healthy males received ketotifen (2 x 1 mg day(-1) p.o.) or placebo for 3 weeks with xamoterol (2 x 200 mg day(-1) p.o.) administered concomitantly during the last 2 weeks. 'l1-adrenoceptor mediated responses were assessed as exercise-induced tachycardia and isoprenaline-induced shortening of heart rate corrected electromechanical systole (QS2c); isoprenaline-induced tachycardia was measured as a mixed beta1-/beta2-adrenoceptor-mediated effect. RESULTS: The first dose of xamoterol significantly increased resting heart rate and systolic blood pressure and significantly shortened QS2c. The last dose of xamoterol after 2 weeks of treatment still produced the same responses. Ketotifen did not influence these effects of xamoterol on resting haemodynamics. The first dose of xamoterol caused a rightward shift of the exercise- and isoprenaline-induced tachycardia (mean dose ratios+/-s.e.mean: 1.20+/-0.05 and 2.46+/-0.23) and the isoprenaline-evoked shortening of QS2c (dose ratio 3.59+/-0.68). This rightward shift was even more pronounced after 2 weeks xamoterol treatment. This additional rightward shift after 2 weeks of xamoterol was not affected by ketotifen (mean difference (95% CI) of log transformed dose ratios between placebo and ketotifen: exercise tachycardia 0.001 (-0.03; 0.04); isoprenaline tachycardia 0.03 (-0.15; 0.21); isoprenaline induced shortening of QS2c 0.13 (-0.22; 0.48)). CONCLUSIONS: In humans xamoterol is a partial beta1-adrenoceptor agonist with positive chrono- and inotropic effects at rest and antagonistic properties under conditions of beta-adrenoceptor stimulation. These effects were well maintained after chronic dosing with no signs of beta1-adrenoceptor desensitization. Ketotifen does not change the beta-adrenoceptor mediated responses of xamoterol after chronic dosing.  (+info)

LocaLisa: new technique for real-time 3-dimensional localization of regular intracardiac electrodes. (4/1171)

BACKGROUND: Estimation of the 3-dimensional (3D) position of ablation electrodes from fluoroscopic images is inadequate if a systematic lesion pattern is required in the treatment of complex arrhythmogenic substrates. METHODS AND RESULTS: We developed a new technique for online 3D localization of intracardiac electrodes. Regular catheter electrodes are used as sensors for a high-frequency transthoracic electrical field, which is applied via standard skin electrodes. We investigated localization accuracy within the right atrium, right ventricle, and left ventricle by comparing measured and true interelectrode distances of a decapolar catheter. Long-term stability was analyzed by localization of the most proximal His bundle before and after slow pathway ablation. Electrogram recordings were unaffected by the applied electrical field. Localization data from 3 catheter positions, widely distributed within the right atrium, right ventricle, or left ventricle, were analyzed in 10 patients per group. The relationship between measured and true electrode positions was highly linear, with an average correlation coefficient of 0.996, 0.997, and 0.999 for the right atrium, right ventricle, and left ventricle, respectively. Localization accuracy was better than 2 mm, with an additional scaling error of 8% to 14%. After 2 hours, localization of the proximal His bundle was reproducible within 1.4+/-1.1 mm. CONCLUSIONS: This new technique enables accurate and reproducible real-time localization of electrode positions in cardiac mapping and ablation procedures. Its application does not distort the quality of electrograms and can be applied to any electrode catheter.  (+info)

Effects of pacing-induced and balloon coronary occlusion ischemia on left atrial function in patients with coronary artery disease. (5/1171)

OBJECTIVES: The aim of this study was to compare left atrial (LA) function in 16 patients with distal left anterior descending (LAD) and in 16 patients with proximal left circumflex (LCx) coronary artery stenosis at rest and immediately after pacing-induced tachycardia (LAD-pacing [P] and LCx-P) or coronary occlusion (LAD-CO and LCx-CO). BACKGROUND: During left ventricular (LV) ischemia, compensatory augmentation of LA contraction enhances LV filling and performance. The left atrium is supplied predominantly by branches arising from the LCx. Therefore, we hypothesized that one mechanism for the loss of atrial contraction may be ischemic LA dysfunction. METHODS: Left ventricular and LA pressure-area relations were derived from simultaneous double-tip micromanometer pressure recordings and automatic boundary detection echocardiograms. RESULTS: Immediately after pacing or after coronary occlusion, LV end-diastolic pressure, LV relaxation, LA mean pressure and LV stiffness significantly increased in all patients. However, the area of the A loop of the LA pressure-area relation, representing the LA pump function, significantly decreased in groups LCx-P and LCx-CO (from 14+/-3 to 9+/-2, and from 16+/-4 to 9+/-2 mm Hg.cm2, respectively, p < 0.05), whereas it increased in groups LAD-P and LAD-CO (from 12+/-3 to 54+/-10, and from 16+/-3 to 49+/-8 mm Hg.cm2, respectively, p < 0.001). CONCLUSIONS: In patients with LAD stenosis, LV supply or demand ischemia is associated with enhanced LA pump function. However, in patients with proximal LCx stenosis who develop the same type and degree of ischemia, LA branches might have been affected, rendering the LA ischemic and unable to increase its booster pump function.  (+info)

Frequency of arrhythmias and other cardiac abnormalities in fulminant hepatic failure. (6/1171)

In a series of 106 patients with fulminant hepatic failure and grade 4 encephalopathy, cardiac arrhythmias and other abnormalities occurred in 92 per cent. The most common was sinus tachycardia (75%) and this was the only abnormality in 22 per cent of the patients. Sudden cardiac arrest occurred in 25 per cent, various ectopic beats in 20 per cent, and heart block or bradycardia in 18 per cent. Other electrocardiographic abnormalities, mostly of the T wave and ST segment, were found in 31 per cent. Cardiac and respiratory arrests were usually unrelated to each other and both frequently occurred without warning. Only 7 out of 71 patients with arrhythmias other than sinus tachycardia survived, compared with 15 out of 31 patients without them (P less than 0-005). During the latter part of the series when an arrhythmia computer was used to monitor 38 patients, it was shown that significantly lower arterial oxygen levels occurred in those with arrhythmias, other than sinus tachycardia, than in those without. They were also found to be more acidotic and hyperkalaemic, and a higher number required dialysis and ventilation. Macroscopical cardiac abnormalities including scattered petechial haemorrhages, small pericardial effusions, and fatty, pale, and flabby ventricles, were found at necropsy in 64 per cent of the patients examined. Combinations of these macroscopical abnormalities occurred, particularly in the paracetamol overdose group. Another necropsy finding of possible significance in the pathogenesis of arrhythmias was cerebral oedema, present in 48 per cent of the patients examined, and often associated with coning of the brain stem. However, 7 of the 16 patients who suffered asystolic cardiac arrests had no macroscopical abnormality of either heart or brain. In the management of patients with fulminant hepatic failure continuous cardiac monitoring is essential. Correction of the biochemical and coagulation defects may decrease the frequency of arrhythmias but studies of the mechanism and control of cerebral oedema and its relation to cardiovascular function are urgently needed.  (+info)

Sino-aortic denervation augments the increase in blood pressure seen during paradoxical sleep in the rat. (7/1171)

Using a computer assisted telemetric system, we have re-examined the effect of sino-aortic denervation (SAD) on the changes in arterial blood pressure (AP) and heart rate (HR) during sleep in the rat suitably recovered from the operation. Eight 1 hourly polygraphic recordings were performed 4 weeks after the initial SAD surgery. In the SAD rats, the increase in AP during paradoxical sleep (PS) was much larger than that in sham-operated rats. HR in the SAD rats increased on-going from slow-wave sleep to PS, but it showed no change in sham-operated rats. The present study suggests that chronic SAD causes the enhanced AP increase during PS concomitantly with the persistent hypertension and tachycardia across sleep-wake states.  (+info)

Aryl propanolamines: comparison of activity at human beta3 receptors, rat beta3 receptors and rat atrial receptors mediating tachycardia. (8/1171)

1. The in vitro activity of four aryl propanolamines was compared to two prototypic beta3 receptor agonists, CGP 12177 and CL316243 at the human beta3 receptor, the rat beta3 receptor in the stomach fundus and receptors mediating atrial tachycardia. 2. L-739,574 was the most potent (EC50 = 9 nM) and selective agonist at the human beta3 receptor with high maximal response (74% of the maximal response to isoproterenol). 3. A phenol-biaryl ether analogue possessed modest affinity for the human beta3 receptor (EC50 = 246 nM), but was highly efficacious with a maximal response 82% of the maximal response to isoproterenol. The other derivatives were intermediate in potency with low maximal responses. 4. These agonists at the human beta3 receptor did not activate the rat beta3 receptor in the rat stomach fundus. In fact, the aryl propanolamines (10(-6) M) inhibited CL316243-induced activation of the rat beta3 receptor. Thus, agonist activity at the human beta3 receptor translated into antagonist activity at the rat beta3 receptor. 5. L739,574 and the phenol biaryl ether increased heart rate via beta1 receptors. 6. Although CGP12177 produced atrial tachycardia, neither the indole sulphonamide nor biphenyl biaryl ether did, although both had high affinity for the human beta3 receptor. Thus, the atrial tachycardic receptor was not identical to the human beta3 receptor. 7. These studies (a) characterized four aryl propanolamines with high affinity at the human beta3 receptor, (b) found that they were antagonists at the rat beta3 receptor, an observation with profound implications for in vivo rat data, and (c) established that the rodent atrial non-beta1, beta2 or beta3 tachycardic receptor was also unrelated to the human beta3 receptor.  (+info)

Tachycardia - WikipediaTachycardia - Wikipedia
Tachycardia, also called tachyarrhythmia, is a heart rate that exceeds the normal resting rate. In general, a resting heart rate over 100 beats per minute is accepted as tachycardia in adults. Heart rates above the resting rate may be normal (such as with exercise) or abnormal (such as with electrical problems within the heart). The upper threshold of a normal human resting heart rate is based on age. Cutoff values for tachycardia in different age groups are fairly well standardized; typical cutoffs are listed below: 1-2 days: Tachycardia > 159 beats per minute (bpm) 3-6 days: Tachycardia >166 bpm 1-3 weeks: Tachycardia >182 bpm 1-2 months: Tachycardia >179 bpm 3-5 months: Tachycardia >186 bpm 6-11 months: Tachycardia >169 bpm 1-2 years: Tachycardia >151 bpm 3-4 years: Tachycardia >137 bpm 5-7 years: Tachycardia >133 bpm 8-11 years: Tachycardia >130 bpm 12-15 years: Tachycardia >119 bpm >15 years - adult: Tachycardia >100 bpm Heart rate is considered in the context of the prevailing clinical ...
Mechanisms of Altered Excitation-Contraction Coupling in Canine Tachycardia-Induced Heart Failure, I | Circulation ResearchMechanisms of Altered Excitation-Contraction Coupling in Canine Tachycardia-Induced Heart Failure, I | Circulation Research
Mechanistic studies of human heart failure are complicated by the prolonged time course of development of the disease, the technical challenges of isolating cardiac tissue or cells from explanted hearts, the inability to investigate the early time course of cellular alterations, and the lack of control over experimental conditions. Thus, it is fortunate that the canine tachycardia-induced heart failure model so closely reproduces the known hemodynamic and ionic changes that have been identified in human hearts. The present findings indicate that, in addition to the electrophysiological changes noted in earlier studies, significant alterations in Ca2+ handling occur in isolated myocytes from failing hearts, following the general pattern of human studies. Through biochemical and functional measurements in the same hearts, we have found strong evidence in support of the hypothesis that the induction of heart failure triggers a shift in the balance of cytosolic Ca2+ extrusion mechanisms from SR Ca2+ ...
Successful Ablation for Atrial Tachycardia Originated from Sinus Venosa with Tachycardia-Induced Cardiomyopathy - pdf descargarSuccessful Ablation for Atrial Tachycardia Originated from Sinus Venosa with Tachycardia-Induced Cardiomyopathy - pdf descargar
Successful Ablation for Atrial Tachycardia Originated from Sinus Venosa with Tachycardia-Induced Cardiomyopathy. . Biblioteca virtual para leer y descargar libros, documentos, trabajos y tesis universitarias en PDF. Material universiario, documentación y tareas realizadas por universitarios en nuestra biblioteca. Para descargar gratis y para leer online.
TachycardiaTachycardia
... refers to a condition of an irregularly rapid beating of the heart. Tachycardia is defined as a heart rate greater than 100 beats per minute, while a normal heart beats is measured at 72 beats per minute. Tachycardia occurs when the electrical impulses that coordinate heartbeats do not function properly and cause the heart to beat irregularly fast. In some situations, an increased heart rate can be normal, such as during exercise and stress. However, depending on the extent of the Tachycardia and the overall health of the patient, tachycardia may be dangerous and require serious medical attention.. If the heart rate is abnormally increased for an extended period of time, oxygen and carbon dioxide levels in the bloodstream can be dramatically altered. Tachycardia can cause stress on the heart because the heart muscle is forced to take less time to relax between each contraction. Patients with Tachycardia may often feel out of breath because the faster the heart beats, the more oxygen ...
Tachycardia in Post-Infarction Hearts: Insights from 3D Image-Based Ventricular Models<...Tachycardia in Post-Infarction Hearts: Insights from 3D Image-Based Ventricular Models<...
TY - JOUR. T1 - Tachycardia in Post-Infarction Hearts. T2 - Insights from 3D Image-Based Ventricular Models. AU - Arevalo, Hermenegild. AU - Plank, Gernot. AU - Helm, Patrick. AU - Halperin, Henry R. AU - Trayanova, Natalia. PY - 2013/7/2. Y1 - 2013/7/2. N2 - Ventricular tachycardia, a life-threatening regular and repetitive fast heart rhythm, frequently occurs in the setting of myocardial infarction. Recently, the peri-infarct zones surrounding the necrotic scar (termed gray zones) have been shown to correlate with ventricular tachycardia inducibility. However, it remains unknown how the latter is determined by gray zone distribution and size. The goal of this study is to examine how tachycardia circuits are maintained in the infarcted heart and to explore the relationship between the tachycardia organizing centers and the infarct gray zone size and degree of heterogeneity. To achieve the goals of the study, we employ a sophisticated high-resolution electrophysiological model of the infarcted ...
Patente US6434424 - Regularization of ventricular rate during atrial tachyarrhythmia - Google PatentesPatente US6434424 - Regularization of ventricular rate during atrial tachyarrhythmia - Google Patentes
A pacing system provided with a mode switching feature and ventricular rate regularization (VRR) function adapted to stabilize or regularize ventricular heart rate during chronic or paroxysmal atrial tachyarrhythmia. In a preferred embodiment, the pacing system nominally operates in an atrial synchronized pacing mode such as DDD or DDDR pacing mode. In response to detection of atrial rhythm characteristics consistent with an atrial tachyarrhythmia, e.g., atrial fibrillation, a mode switch into a non-atrial synchronized, ventricular rate regularization pacing mode, e.g. DDIR or VDIR pacing mode, is made. If the VRR function is programmed on, the ventricular pacing rate based upon a rate responsive sensor derived ventricular pacing rate modulated on a beat by beat basis by preceding intrinsic or paced ventricular events, the stability of the intrinsic ventricular heart rate, and any atrial pace events to regularize the ventricular pacing rate. The pacing system may also be permanently programmed to the
Abdominal Tenderness and Tachycardia: 32 CausesAbdominal Tenderness and Tachycardia: 32 Causes
The freeMD virtual doctor has found 32 conditions that can cause Abdominal Tenderness and Tachycardia. There are 6 common conditions that can cause Abdominal Tenderness and Tachycardia. There are 4 somewhat common conditions that can cause Abdominal Tenderness and Tachycardia. There are 7 uncommon conditions that can cause Abdominal Tenderness and Tachycardia. There are 15 rare conditions that can cause Abdominal Tenderness and Tachycardia.
Tachycardia due to MedicationsTachycardia due to Medications
This page includes the following topics and synonyms: Tachycardia due to Medications, Drug Induced Tachycardia, Toxin Induced Tachycardia.
Tachycardia-Induced Cardiomyopathy: A Review of Animal Models and Clinical Studies | JACC: Journal of the American College of...Tachycardia-Induced Cardiomyopathy: A Review of Animal Models and Clinical Studies | JACC: Journal of the American College of...
The study of conversion of AF to sinus rhythm provides information on changes associated with the resumption of atrial contraction and AV synchrony leading to a regularized, rate-controlled ventricular response. Previous studies have documented improvement in cardiac output ([75]) and cardiopulmonary exercise testing variables of ventricular rate control, maximal oxygen uptake and anaerobic threshold ([76, 77]) after cardioversion to sinus rhythm. More recently, a small, prospective study of patients with a diagnosis of "idiopathic" dilated cardiomyopathy and chronic AF reported a significant increase in left ventricular ejection fraction after pharmacologic or electrical cardioversion to sinus rhythm ([78]). Whether improvement in cardiac function was due to restoration of atrial systolic function and AV synchrony versus reversal of an underlying cardiomyopathy associated with rapid and irregular ventricular rates during AF was not resolved by this study. Subsequent study of atrial and ...
Best Tachycardia Doctor in Chennai, Tachycardia Doctors | CredihealthBest Tachycardia Doctor in Chennai, Tachycardia Doctors | Credihealth
Find the best tachycardia doctors in Chennai. Get guidance from medical experts to select tachycardia specialist in Chennai from trusted hospitals - credihealth.com
A 12 year old with Wide Complex TachycardiaA 12 year old with Wide Complex Tachycardia
The rhythm is a regular WCT ( = Wide-Complex Tachycardia) at ~ 210/minute without sign of atrial activity. Dr. Smith has reviewed the principal differential diagnosis. I dont think QRS duration is particularly helpful here, given that the patient is a 12-year old child (for whom normal QRS duration is a bit less than in adults). So the QRS is definitely wide, though not excessively so - but probably overlaps most entities to be considered. What strikes me - is the obvious suggestion of bifascicular block (RBBB/LAHB), though of atypical morphology. In an otherwise healthy child (who does not have congenital heart disease or other structural heart abnormality) - one would expect a more typical RBBB morphology with a clear triphasic appearance with taller right-rabbit ear in lead V1, instead of the atypical picture we see here in V1,V2,V3. Considering this and integrating it with the important Learning Points Dr. Smith emphasizes (ie, that fascicular VT often occurs in otherwise normal hearts) - ...
Dr. Smiths ECG Blog: A very fast narrow complex tachycardia in an InfantDr. Smith's ECG Blog: A very fast narrow complex tachycardia in an Infant
Most of us (assuming no pediatric cardiologists are listening) do not see a large number of pediatric arrhythmias (certainly far less than weve all seen in adults). As a result - level of comfort in both diagnosis and management tends to be less than that attained with adult arrhythmias. As wonderfully emphasized by Dr. Smith by the 2 pediatric arrhythmia examples shown here - accurate determination of rate is CRITICAL in the assessment of pediatric arrhythmias. For this determination - I have found use of the "Every-other-Beat" (or sometimes, "Every-third-Beat") method a user-friendly way to rapidly and accurate determine heart rate for any regular fast rhythm. While most of us recall, "300- 150 - 100 - 75 - 60 - 50" as the answers for rate calculation when the R-R interval of a regular tachycardia is 1, 2, 3, 4, 5 or 6 large boxes, respectively - this becomes problematic with very fast rates. Thats because small differences in measurements may translate into significant under- or ...
US7295873B1 - Anti-tachycardia pacing method and apparatus for multi-chamber pacing - Google PatentsUS7295873B1 - Anti-tachycardia pacing method and apparatus for multi-chamber pacing - Google Patents
Improved methods and devices perform tachycardia detection and anti-tachycardia pacing (ATP) to convert a tachycardia (e.g., VT or AT) to normal sinus rhythm. According to one embodiment, an anti-tachycardia pacing method includes sensing, during sinus rhythm, first and second cardiac signals at first and second sites, respectively, in a patients heart. The first and second sites include left and right ventricles or left and right atria, for example. The method further includes sensing third and fourth cardiac signals at the first and second sites, respectively, during a tachycardia (e.g., ventricular tachycardia or atrial tachycardia). The cardiac signals are processed to provide respective values. One or more anti-tachycardia pacing pulses are delivered at the site closest to the reentrant circuit based on a comparison of a first ratio of the first and third values and a second ratio of the second and fourth values. Unipolar sensing of the cardiac signals may be employed by, for example,
Tachycardia - Tachyarrhythmias Summary Report | CureHunterTachycardia - Tachyarrhythmias Summary Report | CureHunter
Tachycardia: Abnormally rapid heartbeat, usually with a HEART RATE above 100 beats per minute for adults. Tachycardia accompanied by disturbance in the cardiac depolarization (cardiac arrhythmia) is called tachyarrhythmia.
Tachycardia Arrhythmia | Cardiology | Mercy HealthTachycardia Arrhythmia | Cardiology | Mercy Health
What is tachycardia arrhythmia? Learn about tachycardia arrhythmia (fast heartbeat), including causes, risk factors, symptoms, diagnosis and treatment from the cardiology experts at Mercy Health.
Dr. Smiths ECG Blog: Tachycardia, Dehydration, and New ST Elevation in a 20-something, then a Surprise.Dr. Smith's ECG Blog: Tachycardia, Dehydration, and New ST Elevation in a 20-something, then a Surprise.
Fascinating case with a number of interesting insights to learn. Diagnosis of alternate preexcitation on the last tracing is subtle, but important to recognize. The 2 other thoughts I considered in my differential diagnosis for this last ECG were: i) electrical alternans; and ii) ventricular bigeminy with fusion from end-diastolic PVCs - but neither "fit" (thats because we see DELTA waves in several leads on this tracing that provide the diagnosis). Otherwise - the T wave peaking we see on the tachycardia ECGs is extremely interesting. I have often seen T wave peaking with tachycardia (and on stress tests) in otherwise healthy young adults - but I dont expect ST elevation. In addition, this patient was apparently markedly dehydrated on each occasion when the ECG was done. Hyperkalemia (perhaps secondary to volume contraction) not uncommonly leads to T wave peaking with recurrent episodes, but again supposedly not associated with ST elevation (Wonder what serum K+ values were in this patient ...
Everything you need to know about tachycardiaEverything you need to know about tachycardia
Tachycardia refers to a fast resting heart rate, usually over 100 beats per minute. Tachycardia can be dangerous, depending on its underlying cause and on
Most recent papers with the keyword wide complex tachycardia | Read by QxMDMost recent papers with the keyword wide complex tachycardia | Read by QxMD
BACKGROUND: Catheter ablation (CA) therapy is the first choice treatment in adults with heart rhythm disturbances. The arrhythmias in adults are mainly conditioned by coronary disease. Etiology of arrhythmias in children is mostly associated with inherited heart disorders. According to the current guidelines the CA is widely used in children, indicating the need to make it more achievable in pediatric population. AIM: To assess efficacy and safety of CA in children with different types of arrhythmias on the initial learning curve at newly built Ablation Center in The Independent Pediatric Hospital of Medical University of Warsaw ...
Can Tachycardia Be Dangerous? - CanIdoit.orgCan Tachycardia Be Dangerous? - CanIdoit.org
Most arrhythmia including Tachycardia are not serious However, any abnormality associated with the heart can quickly develop into a serious, or even life
5:09 pm5:09 pm
An interesting technicality in the use of the PERC rule to rule out pulmonary embolism is the tachycardia component - it asks not whether the patient is tachycardic at the time of the application of the rule, or whether tachycardia was sustained throughout the emergency department stay, but instead whether the patient had (as described by Jeff Kline in his great review article on PE diagnosis and risk stratification): "3. Pulse ,100 beats/min during entire stay in ED". Meaning, even transient tachycardia may suggest a life-threatening diagnosis, even if it resolves while the patient is in the emergency department, and were probably PERCing out a whole bunch of patients inappropriately, at least according to Kline (who, notably, testifies a whole bunch as an expert witness in cases of missed pulmonary emboli).. I recently had a handful of patients in whom concerning blood pressures were measured and documented, which then resolved when vital signs were re-checked or after a small quantity of ...
Everyday E(B)M: EKG Challenge No. 11 Case Conclusion: Its Wide! Its Fast! Its a Wide Complex Tachycardia!Everyday E(B)M: EKG Challenge No. 11 Case Conclusion: It's Wide! It's Fast! It's a Wide Complex Tachycardia!
You are working one evening in the emergency department when a 60-something year old female is slotted for a room. Her chief complaint? "Fever, weakness, vomiting". Seeing that her triage heart rate was 157, you leave your granola bar where it is and immediately walk into the room to assess her. You see an elderly-appearing female in moderate respiratory distress. Her temperature is 38.2, blood pressure is 125/87, RR is 32, oxygen saturation is 93% on 5L NC. She has a history of a bone-marrow transplant and is chronically immunosuppressed. She endorses poor PO intake and several episodes of emesis over the last few days. She says that she came in today when she developed some shortness of breath as well. She denies any chest pain or palpitations. On exam, her mucous membranes are dry, her abdomen non-tender, and her breath sounds are decreased in the right base. You are a bit disturbed by the looks of her rhythm strip on the monitor so you get a 12-lead EKG ...
Tachycardia | Reston Hospital CenterTachycardia | Reston Hospital Center
Learn more about Tachycardia at Reston Hospital Center DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...
Tachycardia | Grand Strand HealthTachycardia | Grand Strand Health
Learn more about Tachycardia at Grand Strand Medical Center DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...
Tachycardia: Faster Heart Beat, But Less Blood Flow | InfolificTachycardia: Faster Heart Beat, But Less Blood Flow | Infolific
Tachycardia is a condition where a persons heart is beating abnormally fast. When we become more active our heart rate increases to meet the increased dem
Atrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics: Costs for treatment #250319 in Germany | BookingHealthAtrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics: Costs for treatment #250319 in Germany | BookingHealth
Atrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics (costs for program #250319) ✔ University Hospital Halle (Saale) ✔ Department of Cardiology and Angiology (Department of Internal Medicine III) ✔ BookingHealth.com
Atrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics: Costs for treatment #29263 in Germany | BookingHealthAtrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics: Costs for treatment #29263 in Germany | BookingHealth
Atrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics (costs for program #29263) ✔ Academic Hospital Hildesheim ✔ Department of Cardiology, Angiology, Rhythmology and Intensive Care Medicine ✔ BookingHealth.com
Tachycardia - Resources - Mayo ClinicTachycardia - Resources - Mayo Clinic
Tachycardia is a rapid heart rate caused by a problem in the hearts electrical system. Find out more about this common heart rhythm disorder (arrhythmia).
Pacemaker-Mediated Tachycardia Treatment & Management: Medical CarePacemaker-Mediated Tachycardia Treatment & Management: Medical Care
A pacemaker-mediated tachycardia (PMT) can be defined as any condition in which a pacemaker paces the ventricles at rates that are inappropriately fast. This can be due to (1) a rate response setting that is too sensitive, (2) tracking of atrial noise (such as what may occur with electromagnetic interference), (3) inappropriate pacemaker mani...
TachycardiaTachycardia
A heart rate of more than 100 beats per minute (BPM) in adults is called tachycardia. Whats too fast for you may depend on your age and physical condition.
What are the Main Causes of Tachycardia?What are the Main Causes of Tachycardia?
Rapid and abnormal increase in heart rate more than 100 beats a minute in rest is tachycardia. It is caused by abnormal electrical impulses originated in the heart chambers with a physiological or pathological background.
Just Stopping by (update on my pvcs and tachycardia) - Heart & Cardiovascular Disease - HealingWell.com ForumJust Stopping by (update on my pvcs and tachycardia) - Heart & Cardiovascular Disease - HealingWell.com Forum
Its been a while since I been on here, but I must say life is good, I am truly blessed. My pvcs are almost completely gone, I may feel only a couple a week and I havent had a tachycardia attack in over three months, I must say again God is good. I was once afraid to live on my own because if my heart issues, but I recently moved into my own house three weeks ago. I posted a while back about the doctor putting me on Mirtazapine for anxiety and weight gain, it has really helped me alot, I started off at 111lbs, I am now 134lbs ...
Suffering from SVT tachycardia. Prescribed with metroprolol. Is it safe to go for skiing? - Doctors insight on HealthcareMagicSuffering from SVT tachycardia. Prescribed with metroprolol. Is it safe to go for skiing? - Doctor's insight on HealthcareMagic
Question - Suffering from SVT tachycardia. Prescribed with metroprolol. Is it safe to go for skiing?. Ask a Doctor about diagnosis, treatment and medication for Increased heart rate, Ask a Cardiologist
tachycardia - Heart Disease - MedHelptachycardia - Heart Disease - MedHelp
how high can an axiety attact drive ones HR up? to 110, 120, 130?? I fhigh, one will confuse the adrenaline caused high HR that stops in a few minutes with tachycardia. More and more I believe, aft...
What causes tachycardia? | Reference.comWhat causes tachycardia? | Reference.com
A disruption in the normal electrical impulses controlling the pumping action of the heart causes tachycardia, according to Mayo Clinic. Many different factors cause these disruptions and the related...
is it normal after 9 months for arrhythmias (thumping) to appear - Tachycardia Questionis it normal after 9 months for arrhythmias (thumping) to appear - Tachycardia Question
Had cathetor ablation following a 189 heart beat which I could not control (prior to that over a number of years had, say 120 beats - I could stop by holding my breath a few times and it would disappe
Tachycardia with chest pain - Heart Rhythm - MedHelpTachycardia with chest pain - Heart Rhythm - MedHelp
I have been experiencing a fast, pounding heart rate for over a month now. It gets worse when I stand up. I get light headed, very short of breath, dizzy, weak, and feel as if Im going to faint. I wen...
Tachycardia - what should I do? | Phoenix Rising ME / CFS ForumsTachycardia - what should I do? | Phoenix Rising ME / CFS Forums
I have been wearing a chest hr monitor for the last week and have been concerned that I seem to be tachycardiac in the afternoons. My resting heart...
DI-fusion Tachycardia transition during ablation of persistent atrial...DI-fusion Tachycardia transition during ablation of persistent atrial...
DI-fusion, le Dépôt institutionnel numérique de lULB, est loutil de référencementde la production scientifique de lULB.Linterface de recherche DI-fusion permet de consulter les publications des chercheurs de lULB et les thèses qui y ont été défendues.
TachycardiaTachycardia
Psychiatry healthcare professionals gain a thorough knowledge base of psychiatric disorder information to offer the best patient care. Get our FREE app now.
Abnormal Heartbeat- symptoms & Causes - Health & FitnessAbnormal Heartbeat- symptoms & Causes - Health & Fitness
Abnormal heartbeat is a more common problem than people care to understand. Scientifically known as an Arrhythmia or tachycardia, based on the type, abnormal...
Zinadol Side EffectsZinadol Side Effects
Are Zinadol Side Effects Putting Your Health at Risk? | Jan 10, 2018 Check these Zinadol side effect reports: A 41-year-old female patient was diagnosed with NA, treated with ZINADOL and reported blood pressure increased,tachycardia. Dosage: Unk. Patient was hospitalized.
Guess the pathology? - USMLE ForumlandGuess the pathology? - USMLE Forumland
23 y/o male presents to the ER with tachycardia and hypotension after a high speed MVA. http://forum.usmleforumland.com/filedata/fetch?id=305
Use of the signal-averaged electrocardiogram for predicting inducible ventricular tachycardia in patients with unexplained...Use of the signal-averaged electrocardiogram for predicting inducible ventricular tachycardia in patients with unexplained...
Objectives. The purpose of this study was to determine the predictors of electrically induced ventricular tachycardia in a large sample of patients with unexplained syncope and to examine the value of the signal-averaged electrocardiogram (ECG) in those patient subsets with varying pretest probability of ventricular tachycardia.. Background. In patients with unexplained syncope, electrophysiologic study can provide important diagnostic information, such as inducibility of ventricular tachycardia. The signal-averaged ECG can predict inducible ventricular tachycardia, but its utility has not been prospectively studied in a large group of patients with unexplained syncope.. Methods. At six hospitals, 189 consecutive patients with unexplained syncope underwent signal-averaged ECG and electrophysiologic studies.. Results. Ventricular tachycardia was induced in 28 patients (15%). Univariate predictors of ventricular tachycardia included history of previous myocardial infarction, reduced left ...
Frontiers | Neuronal and hormonal perturbations in postural tachycardia syndrome | PhysiologyFrontiers | Neuronal and hormonal perturbations in postural tachycardia syndrome | Physiology
The Postural Tachycardia Syndrome (POTS) is the most common disorder seen in autonomic clinics. Cardinal hemodynamic feature of this chronic and debilitating disorder of orthostatic tolerance is an exaggerated orthostatic tachycardia (≥30 bpm increase in HR with standing) in the absence of orthostatic hypotension. There are multiple pathophysiological mechanisms that underlie POTS. Some patients with POTS have evidence of elevated sympathoneural tone. This hyperadrenergic state is likely a driver of the excessive orthostatic tachycardia. Another common pathophysiological mechanism in POTS is a hypovolemic state. Many POTS patients with a hypovolemic state have been found to have a perturbed renin-angiotensin-aldosterone profile. These include inappropriately low plasma renin activity and aldosterone levels with resultant inadequate renal sodium retention. Some POTS patients have also been found to have elevated plasma angiotensin II (Ang-II) levels, with some studies suggesting problems with
Management and evaluation of wide QRS complex tachycardia in childrenManagement and evaluation of wide QRS complex tachycardia in children
Wide QRS tachycardia may be due to ventricular tachycardia (VT), supraventricular tachycardia (SVT) with aberrant conduction, or atrioventricular reentrant tachycardia (AVRT) with an accessory pathway. Children with wide QRS complex tachycardia may p
Small-fiber neuropathy with cardiac denervation in postural tachycardia syndrome - Haensch - 2014 - Muscle & Nerve - Wiley...Small-fiber neuropathy with cardiac denervation in postural tachycardia syndrome - Haensch - 2014 - Muscle & Nerve - Wiley...
Introduction: Postural tachycardia syndrome (POTS) is a disorder of orthostatic intolerance characterized by excessive tachycardia of unknown etiology. Our objective in this study was to evaluate the correlation between C-fiber involvement as shown by skin biopsy and adrenergic cardiac metaiodobenzylguanadine (MIBG) uptake in POTS patients. Methods: Skin biopsies of 84 patients with POTS were examined by Protein Gene Product 9.5 (PGP9.5) immunohistochemistry and were compared with MIBG myocardial scintigraphy imaging data. Results: Mean intraepidermal nerve fiber (IENF) density was in the lower normal age-adjusted range, 7.2 ± 2.9/mm (normal ≥7/mm), and was slightly below the normal range in 45% of POTS patients. MIBG uptake was reduced in 21% of patients. Low IENF density correlated with reduced cardiac MIBG uptake (r = 0.39, P = 0.001). Conclusions: A subset of neuropathic POTS patients may harbor mild small fiber neuropathy with abnormalities of unmyelinated nerve fibers in the skin ...
Atrial tachycardia - WikipediaAtrial tachycardia - Wikipedia
Atrial tachycardia is a type of heart rhythm problem in which the hearts electrical impulse comes from an ectopic pacemaker (that is, an abnormally located cardiac pacemaker) in the upper chambers (atria) of the heart, rather than from the sinoatrial node, the normal origin of the hearts electrical activity. Atrial tachycardias can exhibit very regular (consistent) heart rates ranging typically from 140 to 220 beats per minute.[1] As with any other form of tachycardia (rapid heart beat), the underlying mechanism can be either the rapid discharge of an abnormal focus, the presence of a ring of cardiac tissue that gives rise to a circle movement (reentry),[2] or a triggered rapid rhythm due to other pathological circumstances (as would be the case with some drug toxicities, such as digoxin toxicity). Atrial tachycardia is a risk factor for atrial fibrillation, as the rapid rhythm can trigger or degrade into the lack of a rhythm.[3] All atrial tachycardias are by definition supraventricular ...
PoTS - Postural Tachycardia SyndromePoTS - Postural Tachycardia Syndrome
Providing information and support about postural orthostatic tachycardia syndrome (PoTS) for sufferers, medical professionals, family, and friends - aiming to raise awareness.
PoTS - Postural Tachycardia SyndromePoTS - Postural Tachycardia Syndrome
Providing information and support about postural orthostatic tachycardia syndrome (PoTS) for sufferers, medical professionals, family, and friends - aiming to raise awareness.
Ventricular tachycardia - WikipediaVentricular tachycardia - Wikipedia
The morphology of the tachycardia depends on its cause and the origin of the re-entry electrical circuit in the heart. In monomorphic ventricular tachycardia, the shape of each heart beat on the ECG looks the same because the impulse is either being generated from increased automaticity of a single point in either the left or the right ventricle, or due to a reentry circuit within the ventricle. The most common cause of monomorphic ventricular tachycardia is scarring of the heart muscle from a previous myocardial infarction (heart attack). This scar cannot conduct electrical activity, so there is a potential circuit around the scar that results in the tachycardia. This is similar to the re-entrant circuits that are the cause of atrial flutter and the re-entrant forms of supraventricular tachycardia. Other rarer congenital causes of monomorphic VT include right ventricular dysplasia, and right and left ventricular outflow tract VT. Polymorphic ventricular tachycardia, on the other hand, is most ...
Psychiatric profile and attention deficits in postural tachycardia syndrome | Journal of Neurology, Neurosurgery & PsychiatryPsychiatric profile and attention deficits in postural tachycardia syndrome | Journal of Neurology, Neurosurgery & Psychiatry
Objectives: Patients with postural tachycardia syndrome (POTS) often appear anxious and report inattention. Patients with POTS were formally assessed for psychiatric disorders and inattention and compared with patients with attention deficit hyperactivity disorder (ADHD) and control subjects.. Methods: Patients with POTS (n = 21), ADHD (n = 18) and normal control subjects (n = 20) were assessed for DSM-IV psychiatric disorders and completed a battery of questionnaires that assessed depression, anxiety and ADHD characteristics.. Results: Patients with POTS did not have an increased prevalence of major depression or anxiety disorders, including panic disorder, compared with the general population. Patients with POTS had mild depression. They scored as moderately anxious on the Beck Anxiety Inventory but did not exhibit a high level of anxiety sensitivity. Patients with POTS scored significantly higher on inattention and ADHD subscales than control subjects. These symptoms were not present during ...
Modern treatment of the ventricular tachycardia | MedparkModern treatment of the ventricular tachycardia | Medpark
What is ventricular tachycardia? Ventricular tachycardia is an arrhythmia represented by rapid heartbeats that originate in the ventricles. During ventricular tachycardia the heart may beat inefficiently and as a result the blood pressure may decrease very much. This type of tachycardia may lead to life-threatening arrhythmias and cardiac arrest. Treatment options for ventricular tachycardias include medications, catheter ablation or placement of an implantable cardioverter-defibrillator (ICD).
Abstract 10393: Factors of Negativity of Electrophysiological Study in Children and Teenagers Complaining of Tachycardia |...Abstract 10393: Factors of Negativity of Electrophysiological Study in Children and Teenagers Complaining of Tachycardia |...
Background: When noninvasive studies remain negative, the diagnosis of unexplained tachycardia in the youth is a dilemma. Invasive electrophysiological study (EPS) requires generally anesthesia and hospitalization in young children. The purpose of the study was to determine the factors of negativity of transesophageal EPS in children and teenagers complaining of tachycardia.. Population: 267 children, aged from 6 to 19 years, mean 15±3, complained of tachycardia and had a normal ECG in sinus rhythm.. Methods: Transesophageal EPS was performed in out-patient clinic without sedation but in presence of parents; EPS consisted of atrial pacing and atrial stimulation with 1 and 2 extrastimuli in control state and after isoproterenol. Long-term Holter monitoring was systematic in patients with negative EPS.. Results: Supraventricular tachycardia (SVT)(AV node re-entrant tachycardia, AV re-entrant tachycardia, atrial tachycardia) was induced in 146 patients (group I) and EPS remained negative in 121 ...
Supraventricular tachycardia in newborns and its association with gastroesophageal reflux disease | Anales de Pediatría ...Supraventricular tachycardia in newborns and its association with gastroesophageal reflux disease | Anales de Pediatría ...
Introduction. Neonatal tachycardia is defined as a resting heart rate (HR) of 182 beats per minute (bpm) when the baby is not crying.1,2 Supraventricular tachycardia (SVT) is the most common tachyarrhythmia in the neonatal period and usually presents with a HR of more than 200bpm and with a narrow QRS complex. In 80% of patients, it originates through a mechanism of retrograde conduction via an accessory pathway between the ventricle and the atrium, with an abrupt onset and termination. In 15% of patients it results from atrial ectopic beats, while 5% of cases correspond to re-entrant nodal tachycardias.3,4 Approximately half of these patients present with heart failure, with suspicion of sepsis in some cases. The tachycardia may be detected in the foetal period, most frequently between weeks 28 and 33 of gestation, possibly manifesting with hydrops.5. The key element in the management of haemodynamically unstable patients with tachycardia is electric cardioversion. However, in stable patients, ...
Patent US5222501 - Methods for the diagnosis and ablation treatment of ventricular tachycardia - Google PatentsPatent US5222501 - Methods for the diagnosis and ablation treatment of ventricular tachycardia - Google Patents
A closed-heart method for treating ventricular tachycardia in a myocardial infarct patient afflicted with ventricular tachycardia is disclosed. The method comprises, first, defining a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) of the patient, and then ablating the thin layer of spared myocardial tissue by a closed-heart procedure with an ablation catheter. Apparatus for carrying out the method is also disclosed. Also disclosed is a method for prognosing the likelihood of ventricular tachycardia occuring in a myocardial infarct patient not previously diagnosed as afflicted with ventricular tachycardia. The method comprises detecting a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) in the patient.
Patent US5323781 - Methods for the diagnosis and ablation treatment of ventricular tachycardia - Google PatentsPatent US5323781 - Methods for the diagnosis and ablation treatment of ventricular tachycardia - Google Patents
A closed-heart method for treating ventricular tachycardia in a myocardial infarct patient afflicted with ventricular tachycardia is disclosed. The method comprises, first, defining a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) of the patient, and then ablating the thin layer of spared myocardial tissue by a closed-heart procedure with an ablation catheter. Apparatus for carrying out the method is also disclosed, Also disclosed is a method for prognosing the likelihood of ventricular tachycardia occuring in a myocardial infarct patient not previously diagnosed as afflicted with ventricular tachycardia. The method comprises detecting a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) in the patient.
STARS PO Box 175 Stratford-upon-Avon WarwickshireSTARS PO Box 175 Stratford-upon-Avon Warwickshire
... CV37 8YD +44 (0) 1789 450 564 [email protected] www.stars.org.uk Postural Tachycardia Syndrome (PoTS) This information sheet is designed for patients who have been diagnosed with postural tachycardia syndrome (PoTS). It contains information on the diagnosis, treatment and management of PoTS. Postural tachycardia syndrome (PoTS) is an abnormality of functioning of the autonomic (involuntary) nervous system. It is defined as an increase in heart rate of over 30 beats per minute (or to higher than 120 beats per minute) (40 bpm in those age 12-19) when standing upright. Typically there is no postural fall in blood pressure, although fainting (syncope) can occur (see below). The symptoms can vary (see below) but mostly involve orthostatic intolerance (the inability to withstand the upright posture) and are often made worse by various daily activities, e.g., modest physical exertion or food. It is a disorder which is slowly being recognised by the ...
Fetal ventricular tachycardia secondary to long QT syndrome treated with maternal intravenous magnesium : case report and...Fetal ventricular tachycardia secondary to long QT syndrome treated with maternal intravenous magnesium : case report and...
Ventricular tachycardia is a very rare fetal arrhythmia accounting for fewer than 2% of fetal tachycardias. We describe a fetus presenting at 30 weeks gestation with ventricular tachycardia at a rate of 220 beats per min and fetal hydrops. The tachycardia was unresponsive to flecainide but was co...
Sinus tachycardia | Article about Sinus tachycardia by The Free DictionarySinus tachycardia | Article about Sinus tachycardia by The Free Dictionary
Looking for Sinus tachycardia? Find out information about Sinus tachycardia. see arrhythmia arrhythmia , disturbance in the rate or rhythm of the heartbeat. Various arrhythmias can be symptoms of serious heart disorders; however,... Explanation of Sinus tachycardia
Auricular tachycardia | definition of auricular tachycardia by Medical dictionaryAuricular tachycardia | definition of auricular tachycardia by Medical dictionary
Looking for online definition of auricular tachycardia in the Medical Dictionary? auricular tachycardia explanation free. What is auricular tachycardia? Meaning of auricular tachycardia medical term. What does auricular tachycardia mean?
A to Z: TachycardiaA to Z: Tachycardia
The heart has its own electrical system that controls the heart rate and contraction of the heart, which pumps blood throughout the body. Normal heart rates vary based on a persons age.. A rapid heart rate can be caused by many different factors. Sometimes tachycardia is the bodys response to a trigger like fever, anxiety, exercise, or pain, just to name a few. It also can be due to an abnormal rhythm in the heart (called a dysrhythmia), such as supraventricular tachycardia (SVT).. Congenital heart abnormalities, high blood pressure, medications, dehydration, caffeine, smoking, and recreational drug use also can cause tachycardia. Tachycardias can lead to dizziness, shortness of breath, chest pain, fainting, and heart palpitations (a sensation of feeling the heart beat fast or irregularly). Some tachycardias are life threatening and can lead to blood clots, causing a stroke, heart failure, and sudden death.. ...
Evaluation of Chronic Orthostatic Intolerance - Full Text View - ClinicalTrials.govEvaluation of Chronic Orthostatic Intolerance - Full Text View - ClinicalTrials.gov
The subjects are patients with COI and adult healthy volunteers of similar age, gender, and body mass.. Participation in this protocol is offered to people 18 years old or older, independently of gender, race, age, ethnicity, religion, or any other demographic or sociopolitical classifications.. The subjects in the second off-site study are family members of the identified proband. Participation in this protocol is offered to people 18 years old or older, independently of gender, race, ethnicity, religion, or any other demographic or sociopolitical classifications.. In the second off-site study, participation is also offered to subjects 10 to 17 years old, provided that appropriate informed consent/assent has been given by the subject and his/her parent or guardian.. EXCLUSION CRITERIA:. Age: Minors younger than 18 years old are excluded, except for the off-site study. Advanced age does not constitute an exclusion criterion. For subjects more than 55 years old, carotid Doppler studies are done, ...
Top Chronic Orthostatic Hypotension Hospitals in Bangalore | CredihealthTop Chronic Orthostatic Hypotension Hospitals in Bangalore | Credihealth
View details of top chronic orthostatic hypotension hospitals in Bangalore. Get guidance from medical experts to select best chronic orthostatic hypotension hospital in Bangalore
Autoimmune Basis for Postural Tachycardia Syndrome (POTS)Autoimmune Basis for Postural Tachycardia Syndrome (POTS)
Researchers discover autoantibodies in POTS patients that may account for increased circulation of norepinephrine and tachycardia upon standing.
Mode of induction of ventricular tachycardia and prognosis in patients with coronary disease: The multicenter unsustained...Mode of induction of ventricular tachycardia and prognosis in patients with coronary disease: The multicenter unsustained...
TY - JOUR. T1 - Mode of induction of ventricular tachycardia and prognosis in patients with coronary disease. T2 - The multicenter unsustained tachycardia trial (MUSTT). AU - Piccini, Jonathan P.. AU - Hafley, Gail E.. AU - Lee, Kerry L.. AU - Fisher, John D.. AU - Josephson, Mark E.. AU - Prystowsky, Eric N.. AU - Buxton, Alfred E.. PY - 2009/8/1. Y1 - 2009/8/1. N2 - Mode of Induction in MUSTT. Introduction: Programmed stimulation is an important prognostic tool in the evaluation of patients with an ejection fraction ≤40% after myocardial infarction. Many believe that ventricular tachycardia (VT) requiring 3 ventricular extrastimuli (VES) for induction is less likely to occur spontaneously and has less predictive value. However, it is unknown whether the mode of VT induction is associated with long-term prognosis. Methods and Results: We analyzed a cohort of 371 patients enrolled in MUSTT who had inducible monomorphic VT and who were not treated with antiarrhythmic drugs or an implantable ...
POTS - Postural Tachycardia SyndromePOTS - Postural Tachycardia Syndrome
POTS is a relaytively common cause of disabilty in young people, characterized by symptoms associated with fast heartbeat (tachycardia) upon standing.
NEWS DetailNEWS Detail
Case report A 31 year old female was admitted to A/E 6 hours after ingestion of 90 bupropion tablets (13.5g), with alcohol. Initially, the patient was conscious but disorientated, GCS 12/15, agitated, tachycardia (124 beats per minute), raised blood pressure (180/96), tremor, dystonia, increased muscle tone with brisk tendon reflexes and clonus. Activated charcoal was administered but the patient had a convulsion and aspirated. She deteriorated further with recurrent seizures, GCS 5/15, BP 120/60 and heart rate 120 beats per minute and required intubation, ventilation and sedation with diazepam. The ECG showed broad complex tachycardia, which reverted to normal sinus rhythm after treatment with adenosine 12mg iv (Figure 1). The patient was ventilated for 2 days during which she remained hypotensive and had extrapyramidal signs. She was extubated after 3 days. Discussion Bupropion, an atypical antidepressant is structurally similar to amphetamine and diethylpropion.2 It was originally approved ...
Cardiomyopathy induced by sinus tachycardia in combat wounded: a case study.Cardiomyopathy induced by sinus tachycardia in combat wounded: a case study.
Tachycardia induced cardiomyopathy is a potentially lethal cause of heart failure generally because of atrial tachycardia and less frequently ventricular tachycardia. We present two cases of Marines with severe traumatic blast injuries secondary to i
Nitric Oxide and Regulation of Heart Rate in Patients With Postural Tachycardia Syndrome and Healthy SubjectsNovelty and...Nitric Oxide and Regulation of Heart Rate in Patients With Postural Tachycardia Syndrome and Healthy SubjectsNovelty and...
There are 2 main new findings in the present study. The first one is that, after removing autonomic influences, inhibition of endogenous NO synthesis with L-NMMA results in significant bradycardia. This effect is not explained by its pressor effect or baroreflex responses, as the same increase in SBP with phenylephrine did not reduce HR during autonomic blockade. These data show that NO has a tonic effect on human HR in vivo, and that this effect is independent of the autonomic control of HR. It is well known that NO is produced within the heart not only by endothelial cells, but also by myocytes.20,21 It has been proposed that NO has a role in the regulation of cardiac function both under normal and pathological conditions.11,22,23 NOS inhibition was previously shown to reduce HR in patients with transplanted, and presumably denervated, hearts.24 Our findings indicate for the first time that endogenous NO tonically regulates HR in healthy subjects. This is also in agreement with studies in ...
Postural tachycardia syndromePostural tachycardia syndrome
Symptomatic falls in blood pressure after standing or eating are a frequent clinical problem, particularly in the elderly. The symptoms are often due to cerebral hypoperfusion and include generalized weakness, dizziness or lightheadedness, visual blu
Arrhythmia | Heart of MelbourneArrhythmia | Heart of Melbourne
Ventricular tachycardia (VT) is a type of regular, fast heart rate that arises from improper electrical activity in the ventricles of the heart.. ​. Ventricular tachycardia may last for only a few seconds, or it can last for much longer. You may feel dizzy or short of breath, or have chest pain.. Although a few seconds may not result in problems, longer periods are dangerous.. ​. Sometimes, ventricular tachycardia can cause your heart to stop (cardiac arrest), which is a life-threatening medical emergency.. ​. Many different conditions can cause ventricular tachycardia. Its important to get a prompt, accurate diagnosis and appropriate care.. ​. The best way to prevent ventricular tachycardia is to treat or eliminate risk factors that may lead to heart disease. If you already have heart disease, follow your treatment plan and a heart-healthy lifestyle.. See your doctor if you have any problems with the heartbeat. In some cases, urgent care is needed. ...
Frequency and predictors of tachycardia-induced cardiomyopathy in patients with persistent atrial flutter. - PubMed - NCBIFrequency and predictors of tachycardia-induced cardiomyopathy in patients with persistent atrial flutter. - PubMed - NCBI
PubMed comprises more than 30 million citations for biomedical literature from MEDLINE, life science journals, and online books. Citations may include links to full-text content from PubMed Central and publisher web sites.
Narrow Hands & Nonsustained Ventricular Tachycardia & Retinal Arteriovenous Nipping: Causes & Reasons - SymptomaNarrow Hands & Nonsustained Ventricular Tachycardia & Retinal Arteriovenous Nipping: Causes & Reasons - Symptoma
Narrow Hands & Nonsustained Ventricular Tachycardia & Retinal Arteriovenous Nipping Symptom Checker: Possible causes include Coronary Atherosclerosis. Check the full list of possible causes and conditions now! Talk to our Chatbot to narrow down your search.
How does rheumatoid arthritis cause tachycardia - Tips and Tricks From DoctorsHow does rheumatoid arthritis cause tachycardia - Tips and Tricks From Doctors
How does rheumatoid arthritis cause tachycardia - How does rheumatoid arthritis cause tachycardia? Rarely. The pain of the joints and tendon are more likely to cause tachycardia then RA directly.
Epidemiology of Symptomatic Arrhythmias - Full Text View - ClinicalTrials.govEpidemiology of Symptomatic Arrhythmias - Full Text View - ClinicalTrials.gov
BACKGROUND:. Paroxysmal arrhythmias are a group of disorders in which sudden abnormalities of the cardiac rhythm occur without warning. Despite the abundant information available from studies on the mechanisms of tachycardias, there was hardly a shred of objective data in 1989 to establish how the occurrence of symptomatic tachycardias was influenced by various mechanisms. In fact, there were very few objective data describing the occurrence of symptomatic tachycardia among the patients who were afflicted with various paroxysmal tachycardias. For example, were patients in normal sinus rhythm likely to remain free of their tachycardia for one day, one week, one month, or longer? The suddenness and apparent unpredictability of attacks of paroxysmal tachycardias have been substantial obstacles to quantitative description of their occurrence. These studies used careful documentation of spontaneous tachycardia to establish the epidemiology of symptomatic arrhythmias.. DESIGN NARRATIVE:. Baseline ...
MicroRNA-328 Contributes to Adverse Electrical Remodeling in Atrial FibrillationClinical Perspective | CirculationMicroRNA-328 Contributes to Adverse Electrical Remodeling in Atrial FibrillationClinical Perspective | Circulation
Sustained atrial tachycardia as a stress, like clinical AF, can modify atrial properties so that AF recurs and maintains itself more readily; the rapid atrial rate of AF is the primary stimulus of the remodeling process, and such a remodeling has also been called atrial tachycardia remodeling.1,2,42,43 Atrial tachycardia remodeling is suggested to contribute to a variety of clinically important phenomena, including the tendency of paroxysmal AF to become persistent, the tendency of AF to recur soon after cardioversion, and the tendency for longer-lasting AF to become refractory to pharmacological cardioversion. Experimentally, AF and atrial tachycardia remodeling can readily be induced by A-TP.3,4,37-39,42 Intensive studies have been done to understand ionic mechanisms for AF and the associated atrial electric remodeling. It has been widely accepted that for the A-TP-induced electric remodeling, diminished ICaL density resulting from expression downregulation and functional impairment of L-type ...
glossary:sinus tachycardiaglossary:sinus tachycardia
Sinus tachycardia is due to rapid firing of a normal structure called the sinoatrial (sinus) node which is the natural pacemaker of the heart. Sinus tachycardia occurs in response to exercise, exertion, excitement, pain, fever, excessive thyroid hormone, low blood oxygen (hypoxia), stimulant drugs (such as caffeine and amphetamines), etc. ...
Ventricular Tachycardia | Reston Hospital CenterVentricular Tachycardia | Reston Hospital Center
Learn more about Ventricular Tachycardia at Reston Hospital Center DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...
Analysis of Complex Electrophysiologic Data | Clinical Electrophysiology Review, 2e | AccessCardiology | McGraw-Hill MedicalAnalysis of Complex Electrophysiologic Data | Clinical Electrophysiology Review, 2e | AccessCardiology | McGraw-Hill Medical
Two-channel rhythm strip recorded from a patient scheduled for electrophysiology study for palpitations. The onset of the tachycardia occurs after the second QRS and the P wave is noted in the following diastole to be of different morphology than the probable initial sinus P wave. The identification of the P wave during tachycardia is facilitated by comparison with the last QRS in the strip that is not followed by a P wave. Careful measurement with calipers (a critical tool of the electrophysiologist) will illustrate that the onset of the tachycardia does not require PR prolongation. In addition, the cycle length varies and during this, the PR stays constant while the apparent RP varies. These findings are most compatible with an atrial tachycardia. Note that most junctional reentrant tachycardias would require some AV delay at the onset and such variable retrograde conduction would be unusual. This information provides a focused starting point to plan electrophysiology study. ...
Patent US5205283 - Method and apparatus for tachyarrhythmia detection and treatment - Google PatentsPatent US5205283 - Method and apparatus for tachyarrhythmia detection and treatment - Google Patents
An implantable cardioverter capable of delivering pacing and cardioversion level therapies. The device distinguishes between stable and unstable ventricular tachyarrhythmias by monitoring the progression of atrial cycle lengths during the detected ventricular tachyarrhythmia. A detected increase in atrial cycle lengths during the early stages of the detected ventricular tachycardia is taken as an indication of hemodynamically unstable ventricular tachycardia.
Limited predictive value of inducible sustained ventricular tachycardia for future occurrence of spontaneous ventricular...Limited predictive value of inducible sustained ventricular tachycardia for future occurrence of spontaneous ventricular...
To evaluate the significance of inducible sustained ventricular tachycardia (VT) in patients with coronary artery disease and relatively preserved cardiac function, 33 patients who met the following criteria were studied; documented nonsustained VT b
Ventricular Tachycardia (VT) | Frankel Cardiovascular Center | Michigan MedicineVentricular Tachycardia (VT) | Frankel Cardiovascular Center | Michigan Medicine
Ventricular tachycardia (VT) is a rapid heart rate that starts in the ventricles. If VT continues untreated, it can cause sudden cardiac death.
Ventricular Tachycardia (VT)Ventricular Tachycardia (VT)
Ventricular tachycardia is a fast, abnormal heart rate that begins in the lower chambers of the heart. It can become life-threatening if it lasts more than a few seconds.
Device diagnosis of pacemaker-mediated tachycardia: true or false?Device diagnosis of pacemaker-mediated tachycardia: true or false?
article{3091001, author = {Barold, S Serge and Stroobandt, Roland and Van Heuverswyn, Frederic}, issn = {0147-8389}, journal = {PACE-PACING AND CLINICAL ELECTROPHYSIOLOGY}, keywords = {pacing,pacemaker-mediated tachycardia,electrocardiogram}, language = {eng}, number = {1}, pages = {116--118}, title = {Device diagnosis of pacemaker-mediated tachycardia: true or false?}, url = {http://dx.doi.org/10.1111/j.1540-8159.2012.03463.x}, volume = {36}, year = {2013 ...
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What causes tachycardia? | Reference.comWhat causes tachycardia? | Reference.com
TachycardiaTachycardia
Tachycardia | Grand Strand HealthTachycardia | Grand Strand Health
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is it normal after 9 months for arrhythmias (thumping) to appear - Tachycardia Questionis it normal after 9 months for arrhythmias (thumping) to appear - Tachycardia Question
Abdominal Tenderness and Tachycardia: 32 CausesAbdominal Tenderness and Tachycardia: 32 Causes
Tachycardia-Induced Cardiomyopathy: A Review of Animal Models and Clinical Studies | JACC: Journal of the American College of...Tachycardia-Induced Cardiomyopathy: A Review of Animal Models and Clinical Studies | JACC: Journal of the American College of...
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Catecholaminergic polymorphic ventricular tachycardia | SpringerLinkCatecholaminergic polymorphic ventricular tachycardia | SpringerLink
Tachycardia: Causes, symptoms, and treatmentsTachycardia: Causes, symptoms, and treatments
Postural tachycardia syndromePostural tachycardia syndrome
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Catecholaminergic polymorphic ventricular tachycardia: MedlinePlus GeneticsCatecholaminergic polymorphic ventricular tachycardia: MedlinePlus Genetics
What are the symptoms of tachycardia?What are the symptoms of tachycardia?
Paroxysmal Supraventricular Tachycardia (PSVT)Paroxysmal Supraventricular Tachycardia (PSVT)
Paroxysmal Supraventricular Tachycardia (PSVT)Paroxysmal Supraventricular Tachycardia (PSVT)
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Infographic: Ventricular Tachycardia - Mayo ClinicInfographic: Ventricular Tachycardia - Mayo Clinic
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SinusAblationArrhythmiaParoxysmal SupraventriSymptoms of tachycardiaComplex tachycardiasConductionCause tachycardiaDiagnosisAbnormalAbnormallyPacemakerVentricular tachycardia inducibilityHeart'sBeatsCauses tachycardiaTachyarrhythmiaMyocardialRhythmHeart DiseaseHyperthyroidismSupraventricular tachycardiasParoxysmalNarrow complex tachycardiaPSVTComplex tachycardiaHeartbeatType of tachycardiaCatecholaminergicEctopicCardiacAVRTAtriaSyncopeTypes of tachycardiaPoTS2016PolymorphicOrthostatic tachycardiaHeart rhythmVentriclesMonomorphic ventricularVentricular tachycardiasCardiomyopathyOccursPostural tachycardiaCPVTPacemaker-mediated tachycardiaForms of ventricular tachycardiaRefersSudden deathPalpitationsEpisodesCause of the tachycardiaFocal atrial tachycardia
Sinus4
Ablation1
  • The findings of this study have important implications for the advancement of improved criteria for stratifying arrhythmia risk in post-infarction patients and for the development of new approaches for determining the ablation targets of infarct-related tachycardia. (elsevier.com)
Arrhythmia7
Paroxysmal Supraventri2
Symptoms of tachycardia1
Complex tachycardias3
Conduction2
Cause tachycardia5
Diagnosis1
Abnormal2
Abnormally1
Pacemaker2
Ventricular tachycardia inducibility1
Heart's2
Beats5
Causes tachycardia2
Tachyarrhythmia1
Myocardial1
Rhythm1
Heart Disease1
Hyperthyroidism1
  • Certain diseases can contribute to tachycardia, including hyperthyroidism and anemia, and tachycardia can be an accompanying symptom of fever in some illnesses. (reference.com)
Supraventricular tachycardias8
Paroxysmal24
Narrow complex tachycardia5
PSVT3
Complex tachycardia5
Heartbeat9
Type of tachycardia3
Catecholaminergic17
Ectopic8
Cardiac13
AVRT1
Atria7
Syncope2
Types of tachycardia2
PoTS13
  • This disorder has been called the postural tachycardia syndrome or POTS [ 1-3 ]. (uptodate.com)
  • Considering taking a vitamin or supplement to treat Postural+Tachycardia+Syndrome+(Pots)? (webmd.com)
  • Below is a list of common natural remedies used to treat or reduce the symptoms of Postural+Tachycardia+Syndrome+(Pots). (webmd.com)
  • Postural tachycardia syndrome (POTS) is a disabling condition that commonly affects otherwise normal young females. (nih.gov)
  • POT syndrome (POTS, postrual orthostatic tachycardia syndrome) is a nervous system disorder that causes lightheadedness and fainting when a person stands up. (medicinenet.com)
  • We report on a series of patients who developed autonomic dysfunction in the form of postural orthostatic tachycardia syndrome (POTS). (nih.gov)
  • A new scientific paper from Prof Newton's group (published this year in the Journal of Internal Medicine ) describes postural orthostatic tachycardia syndrome (POTS), an aspect of autonomic dysfunctionthat can produce substantial disability among otherwise healthy people. (prohealth.com)
  • The current study aimed to investigate the association between vitamin B 12 levels and postural orthostatic tachycardia syndrome (POTS) during adolescence when accelerated myelin synthesis increases the vitamin B 12 need. (aappublications.org)
  • Postural orthostatic tachycardia syndrome (POTS) is one of the most common forms of chronic orthostatic intolerance in the general population. (aappublications.org)
  • Patients with postural tachycardia syndrome (POTS) have exaggerated orthostatic tachycardia often following a viral illness, suggesting autoimmunity may play a pathophysiological role in POTS. (prohealth.com)
  • POTS patients have elevated alpha1AR autoantibodies exerting a partial peripheral antagonist effect resulting in a compensatory sympathoneural activation of alpha1AR for vasoconstriction and concurrent betaAR-mediated tachycardia. (prohealth.com)
  • These findings may explain the increased standing plasma norepinephrine and excessive tachycardia observed in many POTS patients. (prohealth.com)
  • David C. Kem, MD, Autoimmune Basis for Postural Tachycardia Syndrome (POTS). (prohealth.com)
20162
Polymorphic2
Orthostatic tachycardia3
Heart rhythm8
Ventricles3
Monomorphic ventricular3
Ventricular tachycardias1
Cardiomyopathy9
Occurs7
Postural tachycardia2
CPVT1
Pacemaker-mediated tachycardia7
  • The presence of a paced rhythm exactly at the upper rate limit with atrial sensing and exact A-V association warrants evaluation for pacemaker-mediated tachycardia (PMT). (medscape.com)
  • Pacemaker-mediated tachycardia: manufacturer specifics and spectrum of cases. (medscape.com)
  • Tracking of atrial flutter during DDD pacing: another form of pacemaker-mediated tachycardia. (medscape.com)
  • Validation of device algorithm to differentiate pacemaker-mediated tachycardia from tachycardia due to atrial tracking. (medscape.com)
  • Accuracy of the pacemaker-mediated tachycardia algorithm in Boston Scientific devices. (medscape.com)
  • Telemetered ECG tracing with surface lead II (top) and intracardiac electrograms (atrial electrogram [center] and ventricular electrogram [lower]) and marker channel (bottom) showing pacemaker-mediated tachycardia (PMT). (medscape.com)
  • Telemetered ECG tracing showing atrioventricular (AV)-paced rhythm at 60/min after termination of the pacemaker-mediated tachycardia (PMT). (medscape.com)
Forms of ventricular tachycardia1
Refers4
Sudden death4
Palpitations2
Episodes1
Cause of the tachycardia2
  • Sometimes, the medical team may not be able to identify the exact cause of the tachycardia. (medicalnewstoday.com)
  • The aim of treatment is to address the cause of the tachycardia. (medicalnewstoday.com)
Focal atrial tachycardia1
Tachycardia - Heart Disease - MedHelp
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Ventricular Tachycardia (VT)
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CHANGES: Living with Postural Orthostatic Tachycardia Syndrome - YouTube
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