Abnormally rapid heartbeat, usually with a HEART RATE above 100 beats per minute for adults. Tachycardia accompanied by disturbance in the cardiac depolarization (cardiac arrhythmia) is called tachyarrhythmia.
An abnormally rapid ventricular rhythm usually in excess of 150 beats per minute. It is generated within the ventricle below the BUNDLE OF HIS, either as autonomic impulse formation or reentrant impulse conduction. Depending on the etiology, onset of ventricular tachycardia can be paroxysmal (sudden) or nonparoxysmal, its wide QRS complexes can be uniform or polymorphic, and the ventricular beating may be independent of the atrial beating (AV dissociation).
A generic expression for any tachycardia that originates above the BUNDLE OF HIS.
Abnormally rapid heartbeats caused by reentry of atrial impulse into the dual (fast and slow) pathways of ATRIOVENTRICULAR NODE. The common type involves a blocked atrial impulse in the slow pathway which reenters the fast pathway in a retrograde direction and simultaneously conducts to the atria and the ventricles leading to rapid HEART RATE of 150-250 beats per minute.
Abnormally rapid heartbeats with sudden onset and cessation.
Abnormally rapid heartbeats originating from one or more automatic foci (nonsinus pacemakers) in the HEART ATRIUM but away from the SINOATRIAL NODE. Unlike the reentry mechanism, automatic tachycardia speeds up and slows down gradually. The episode is characterized by a HEART RATE between 135 to less than 200 beats per minute and lasting 30 seconds or longer.
Simple rapid heartbeats caused by rapid discharge of impulses from the SINOATRIAL NODE, usually between 100 and 180 beats/min in adults. It is characterized by a gradual onset and termination. Sinus tachycardia is common in infants, young children, and adults during strenuous physical activities.
Removal of tissue with electrical current delivered via electrodes positioned at the distal end of a catheter. Energy sources are commonly direct current (DC-shock) or alternating current at radiofrequencies (usually 750 kHz). The technique is used most often to ablate the AV junction and/or accessory pathways in order to interrupt AV conduction and produce AV block in the treatment of various tachyarrhythmias.
Recording of the moment-to-moment electromotive forces of the HEART as projected onto various sites on the body's surface, delineated as a scalar function of time. The recording is monitored by a tracing on slow moving chart paper or by observing it on a cardioscope, which is a CATHODE RAY TUBE DISPLAY.
A rare form of supraventricular tachycardia caused by automatic, not reentrant, conduction initiated from sites at the atrioventricular junction, but not the ATRIOVENTRICULAR NODE. It usually occurs during myocardial infarction, after heart surgery, or in digitalis intoxication with a HEART RATE ranging from 140 to 250 beats per minute.
A syndrome of ORTHOSTATIC INTOLERANCE combined with excessive upright TACHYCARDIA, and usually without associated ORTHOSTATIC HYPOTENSION. All variants have in common an excessively reduced venous return to the heart (central HYPOVOLEMIA) while upright.
Abnormally rapid heartbeats caused by reentrant conduction over the accessory pathways between the HEART ATRIA and the HEART VENTRICLES. The impulse can also travel in the reverse direction, as in some cases, atrial impulses travel to the ventricles over the accessory pathways and back to the atria over the BUNDLE OF HIS and the ATRIOVENTRICULAR NODE.
Abnormally rapid heartbeats caused by reentry circuit in or around the SINOATRIAL NODE. It is characterized by sudden onset and offset episodes of tachycardia with a HEART RATE of 100-150 beats per minute. The P wave is identical to the sinus P wave but with a longer PR interval.
An impulse-conducting system composed of modified cardiac muscle, having the power of spontaneous rhythmicity and conduction more highly developed than the rest of the heart.
Methods to induce and measure electrical activities at specific sites in the heart to diagnose and treat problems with the heart's electrical system.
Regulation of the rate of contraction of the heart muscles by an artificial pacemaker.
Agents used for the treatment or prevention of cardiac arrhythmias. They may affect the polarization-repolarization phase of the action potential, its excitability or refractoriness, or impulse conduction or membrane responsiveness within cardiac fibers. Anti-arrhythmia agents are often classed into four main groups according to their mechanism of action: sodium channel blockade, beta-adrenergic blockade, repolarization prolongation, or calcium channel blockade.
A small nodular mass of specialized muscle fibers located in the interatrial septum near the opening of the coronary sinus. It gives rise to the atrioventricular bundle of the conduction system of the heart.
Small band of specialized CARDIAC MUSCLE fibers that originates in the ATRIOVENTRICULAR NODE and extends into the membranous part of the interventricular septum. The bundle of His, consisting of the left and the right bundle branches, conducts the electrical impulses to the HEART VENTRICLES in generation of MYOCARDIAL CONTRACTION.
Recording of regional electrophysiological information by analysis of surface potentials to give a complete picture of the effects of the currents from the heart on the body surface. It has been applied to the diagnosis of old inferior myocardial infarction, localization of the bypass pathway in Wolff-Parkinson-White syndrome, recognition of ventricular hypertrophy, estimation of the size of a myocardial infarct, and the effects of different interventions designed to reduce infarct size. The limiting factor at present is the complexity of the recording and analysis, which requires 100 or more electrodes, sophisticated instrumentation, and dedicated personnel. (Braunwald, Heart Disease, 4th ed)
Any disturbances of the normal rhythmic beating of the heart or MYOCARDIAL CONTRACTION. Cardiac arrhythmias can be classified by the abnormalities in HEART RATE, disorders of electrical impulse generation, or impulse conduction.
A form of ventricular pre-excitation characterized by a short PR interval and a long QRS interval with a delta wave. In this syndrome, atrial impulses are abnormally conducted to the HEART VENTRICLES via an ACCESSORY CONDUCTING PATHWAY that is located between the wall of the right or left atria and the ventricles, also known as a BUNDLE OF KENT. The inherited form can be caused by mutation of PRKAG2 gene encoding a gamma-2 regulatory subunit of AMP-activated protein kinase.
A potentially lethal cardiac arrhythmia that is characterized by uncoordinated extremely rapid firing of electrical impulses (400-600/min) in HEART VENTRICLES. Such asynchronous ventricular quivering or fibrillation prevents any effective cardiac output and results in unconsciousness (SYNCOPE). It is one of the major electrocardiographic patterns seen with CARDIAC ARREST.
The number of times the HEART VENTRICLES contract per unit of time, usually per minute.
Implantable devices which continuously monitor the electrical activity of the heart and automatically detect and terminate ventricular tachycardia (TACHYCARDIA, VENTRICULAR) and VENTRICULAR FIBRILLATION. They consist of an impulse generator, batteries, and electrodes.
An antianginal and class III antiarrhythmic drug. It increases the duration of ventricular and atrial muscle action by inhibiting POTASSIUM CHANNELS and VOLTAGE-GATED SODIUM CHANNELS. There is a resulting decrease in heart rate and in vascular resistance.
Method in which prolonged electrocardiographic recordings are made on a portable tape recorder (Holter-type system) or solid-state device ("real-time" system), while the patient undergoes normal daily activities. It is useful in the diagnosis and management of intermittent cardiac arrhythmias and transient myocardial ischemia.
A potent anti-arrhythmia agent, effective in a wide range of ventricular and atrial ARRHYTHMIAS and TACHYCARDIAS.
A type of cardiac arrhythmia with premature contractions of the HEART VENTRICLES. It is characterized by the premature QRS complex on ECG that is of abnormal shape and great duration (generally >129 msec). It is the most common form of all cardiac arrhythmias. Premature ventricular complexes have no clinical significance except in concurrence with heart diseases.
The use of freezing as a special surgical technique to destroy or excise tissue.
The chambers of the heart, to which the BLOOD returns from the circulation.
A class Ia antiarrhythmic drug that is structurally-related to PROCAINE.
The innermost layer of the heart, comprised of endothelial cells.
Rapid, irregular atrial contractions caused by a block of electrical impulse conduction in the right atrium and a reentrant wave front traveling up the inter-atrial septum and down the right atrial free wall or vice versa. Unlike ATRIAL FIBRILLATION which is caused by abnormal impulse generation, typical atrial flutter is caused by abnormal impulse conduction. As in atrial fibrillation, patients with atrial flutter cannot effectively pump blood into the lower chambers of the heart (HEART VENTRICLES).
Impaired conduction of cardiac impulse that can occur anywhere along the conduction pathway, such as between the SINOATRIAL NODE and the right atrium (SA block) or between atria and ventricles (AV block). Heart blocks can be classified by the duration, frequency, or completeness of conduction block. Reversibility depends on the degree of structural or functional defects.
A form of heart block in which the electrical stimulation of HEART VENTRICLES is interrupted at either one of the branches of BUNDLE OF HIS thus preventing the simultaneous depolarization of the two ventricles.
A group of conditions in which HEART VENTRICLE activation by the atrial impulse is faster than the normal impulse conduction from the SINOATRIAL NODE. In these pre-excitation syndromes, atrial impulses often bypass the ATRIOVENTRICULAR NODE delay and travel via ACCESSORY CONDUCTING PATHWAYS connecting the atrium directly to the BUNDLE OF HIS.
Extra impulse-conducting tissue in the heart that creates abnormal impulse-conducting connections between HEART ATRIA and HEART VENTRICLES.
The lower right and left chambers of the heart. The right ventricle pumps venous BLOOD into the LUNGS and the left ventricle pumps oxygenated blood into the systemic arterial circulation.
A group of cardiac arrhythmias in which the cardiac contractions are not initiated at the SINOATRIAL NODE. They include both atrial and ventricular premature beats, and are also known as extra or ectopic heartbeats. Their frequency is increased in heart diseases.
The study of the generation and behavior of electrical charges in living organisms particularly the nervous system and the effects of electricity on living organisms.
Unexpected rapid natural death due to cardiovascular collapse within one hour of initial symptoms. It is usually caused by the worsening of existing heart diseases. The sudden onset of symptoms, such as CHEST PAIN and CARDIAC ARRHYTHMIAS, particularly VENTRICULAR TACHYCARDIA, can lead to the loss of consciousness and cardiac arrest followed by biological death. (from Braunwald's Heart Disease: A Textbook of Cardiovascular Medicine, 7th ed., 2005)
An electrical current applied to the HEART to terminate a disturbance of its rhythm, ARRHYTHMIAS, CARDIAC. (Stedman, 25th ed)
A transient loss of consciousness and postural tone caused by diminished blood flow to the brain (i.e., BRAIN ISCHEMIA). Presyncope refers to the sensation of lightheadedness and loss of strength that precedes a syncopal event or accompanies an incomplete syncope. (From Adams et al., Principles of Neurology, 6th ed, pp367-9)
Recording the locations and measurements of electrical activity in the EPICARDIUM by placing electrodes on the surface of the heart to analyze the patterns of activation and to locate arrhythmogenic sites.
Studies in which individuals or populations are followed to assess the outcome of exposures, procedures, or effects of a characteristic, e.g., occurrence of disease.
Evaluation undertaken to assess the results or consequences of management and procedures used in combating disease in order to determine the efficacy, effectiveness, safety, and practicability of these interventions in individual cases or series.
An adrenergic beta-antagonist that is used in the treatment of life-threatening arrhythmias.
Abnormal cardiac rhythm that is characterized by rapid, uncoordinated firing of electrical impulses in the upper chambers of the heart (HEART ATRIA). In such case, blood cannot be effectively pumped into the lower chambers of the heart (HEART VENTRICLES). It is caused by abnormal impulse generation.
Abnormal accumulation of serous fluid in two or more fetal compartments, such as SKIN; PLEURA; PERICARDIUM; PLACENTA; PERITONEUM; AMNIOTIC FLUID. General fetal EDEMA may be of non-immunologic origin, or of immunologic origin as in the case of ERYTHROBLASTOSIS FETALIS.
A congenital cardiomyopathy that is characterized by infiltration of adipose and fibrous tissue into the RIGHT VENTRICLE wall and loss of myocardial cells. Primary injuries usually are at the free wall of right ventricular and right atria resulting in ventricular and supraventricular arrhythmias.
The return of a sign, symptom, or disease after a remission.
A group of diseases in which the dominant feature is the involvement of the CARDIAC MUSCLE itself. Cardiomyopathies are classified according to their predominant pathophysiological features (DILATED CARDIOMYOPATHY; HYPERTROPHIC CARDIOMYOPATHY; RESTRICTIVE CARDIOMYOPATHY) or their etiological/pathological factors (CARDIOMYOPATHY, ALCOHOLIC; ENDOCARDIAL FIBROELASTOSIS).
The domestic dog, Canis familiaris, comprising about 400 breeds, of the carnivore family CANIDAE. They are worldwide in distribution and live in association with people. (Walker's Mammals of the World, 5th ed, p1065)
A device designed to stimulate, by electric impulses, contraction of the heart muscles. It may be temporary (external) or permanent (internal or internal-external).
A conical fibro-serous sac surrounding the HEART and the roots of the great vessels (AORTA; VENAE CAVAE; PULMONARY ARTERY). Pericardium consists of two sacs: the outer fibrous pericardium and the inner serous pericardium. The latter consists of an outer parietal layer facing the fibrous pericardium, and an inner visceral layer (epicardium) resting next to the heart, and a pericardial cavity between these two layers.
Drugs that bind to but do not activate beta-adrenergic receptors thereby blocking the actions of beta-adrenergic agonists. Adrenergic beta-antagonists are used for treatment of hypertension, cardiac arrhythmias, angina pectoris, glaucoma, migraine headaches, and anxiety.
The study of the electrical activity and characteristics of the HEART; MYOCARDIUM; and CARDIOMYOCYTES.
Elements of limited time intervals, contributing to particular results or situations.
An antiarrhythmia agent that is particularly effective in ventricular arrhythmias. It also has weak beta-blocking activity.
Cardiac arrhythmias that are characterized by excessively slow HEART RATE, usually below 50 beats per minute in human adults. They can be classified broadly into SINOATRIAL NODE dysfunction and ATRIOVENTRICULAR BLOCK.
A form of ventricular pre-excitation characterized by a normal PR interval and a long QRS interval with an initial slow deflection (delta wave). In this syndrome, the atrial impulse travel to the ventricle via the MAHAIM FIBERS which connect ATRIOVENTRICULAR NODE directly to the right ventricle wall (NODOVENTRICULAR ACCESSORY PATHWAY) or to the RIGHT BUNDLE BRANCH OF HIS (nodofascicular accessory pathway).
A calcium channel blocker that is a class IV anti-arrhythmia agent.
The hollow, muscular organ that maintains the circulation of the blood.
Impaired impulse conduction from HEART ATRIA to HEART VENTRICLES. AV block can mean delayed or completely blocked impulse conduction.
A widely used non-cardioselective beta-adrenergic antagonist. Propranolol has been used for MYOCARDIAL INFARCTION; ARRHYTHMIA; ANGINA PECTORIS; HYPERTENSION; HYPERTHYROIDISM; MIGRAINE; PHEOCHROMOCYTOMA; and ANXIETY but adverse effects instigate replacement by newer drugs.
The movement and the forces involved in the movement of the blood through the CARDIOVASCULAR SYSTEM.
NECROSIS of the MYOCARDIUM caused by an obstruction of the blood supply to the heart (CORONARY CIRCULATION).
A cardiotonic glycoside obtained mainly from Digitalis lanata; it consists of three sugars and the aglycone DIGOXIGENIN. Digoxin has positive inotropic and negative chronotropic activity. It is used to control ventricular rate in ATRIAL FIBRILLATION and in the management of congestive heart failure with atrial fibrillation. Its use in congestive heart failure and sinus rhythm is less certain. The margin between toxic and therapeutic doses is small. (From Martindale, The Extra Pharmacopoeia, 30th ed, p666)
The small mass of modified cardiac muscle fibers located at the junction of the superior vena cava (VENA CAVA, SUPERIOR) and right atrium. Contraction impulses probably start in this node, spread over the atrium (HEART ATRIUM) and are then transmitted by the atrioventricular bundle (BUNDLE OF HIS) to the ventricle (HEART VENTRICLE).
Antiarrhythmic agent pharmacologically similar to LIDOCAINE. It may have some anticonvulsant properties.
A standard and widely accepted diagnostic test used to identify patients who have a vasodepressive and/or cardioinhibitory response as a cause of syncope. (From Braunwald, Heart Disease, 7th ed)
Studies used to test etiologic hypotheses in which inferences about an exposure to putative causal factors are derived from data relating to characteristics of persons under study or to events or experiences in their past. The essential feature is that some of the persons under study have the disease or outcome of interest and their characteristics are compared with those of unaffected persons.
A condition in which HEART VENTRICLES exhibit impaired function.
Procedures using an electrically heated wire or scalpel to treat hemorrhage (e.g., bleeding ulcers) and to ablate tumors, mucosal lesions, and refractory arrhythmias. It is different from ELECTROSURGERY which is used more for cutting tissue than destroying and in which the patient is part of the electric circuit.
Acidic protein found in SARCOPLASMIC RETICULUM that binds calcium to the extent of 700-900 nmoles/mg. It plays the role of sequestering calcium transported to the interior of the intracellular vesicle.
Ultrasonic recording of the size, motion, and composition of the heart and surrounding tissues. The standard approach is transthoracic.
Procedures in which placement of CARDIAC CATHETERS is performed for therapeutic or diagnostic procedures.
The abrupt cessation of all vital bodily functions, manifested by the permanent loss of total cerebral, respiratory, and cardiovascular functions.
Isopropyl analog of EPINEPHRINE; beta-sympathomimetic that acts on the heart, bronchi, skeletal muscle, alimentary tract, etc. It is used mainly as bronchodilator and heart stimulant.
The hemodynamic and electrophysiological action of the RIGHT ATRIUM.
Pathophysiological conditions of the FETUS in the UTERUS. Some fetal diseases may be treated with FETAL THERAPIES.
A form of CARDIAC MUSCLE disease that is characterized by ventricular dilation, VENTRICULAR DYSFUNCTION, and HEART FAILURE. Risk factors include SMOKING; ALCOHOL DRINKING; HYPERTENSION; INFECTION; PREGNANCY; and mutations in the LMNA gene encoding LAMIN TYPE A, a NUCLEAR LAMINA protein.
The period of time following the triggering of an ACTION POTENTIAL when the CELL MEMBRANE has changed to an unexcitable state and is gradually restored to the resting (excitable) state. During the absolute refractory period no other stimulus can trigger a response. This is followed by the relative refractory period during which the cell gradually becomes more excitable and the stronger impulse that is required to illicit a response gradually lessens to that required during the resting state.
Observation of a population for a sufficient number of persons over a sufficient number of years to generate incidence or mortality rates subsequent to the selection of the study group.
The fibrous tissue that replaces normal tissue during the process of WOUND HEALING.
Cessation of heart beat or MYOCARDIAL CONTRACTION. If it is treated within a few minutes, heart arrest can be reversed in most cases to normal cardiac rhythm and effective circulation.
A condition that is characterized by episodes of fainting (SYNCOPE) and varying degree of ventricular arrhythmia as indicated by the prolonged QT interval. The inherited forms are caused by mutation of genes encoding cardiac ion channel proteins. The two major forms are ROMANO-WARD SYNDROME and JERVELL-LANGE NIELSEN SYNDROME.
A type of cardiac arrhythmia with premature atrial contractions or beats caused by signals originating from ectopic atrial sites. The ectopic signals may or may not conduct to the HEART VENTRICLES. Atrial premature complexes are characterized by premature P waves on ECG which are different in configuration from the P waves generated by the normal pacemaker complex in the SINOATRIAL NODE.
The veins that return the oxygenated blood from the lungs to the left atrium of the heart.
A malignant form of polymorphic ventricular tachycardia that is characterized by HEART RATE between 200 and 250 beats per minute, and QRS complexes with changing amplitude and twisting of the points. The term also describes the syndrome of tachycardia with prolonged ventricular repolarization, long QT intervals exceeding 500 milliseconds or BRADYCARDIA. Torsades de pointes may be self-limited or may progress to VENTRICULAR FIBRILLATION.
An autosomal dominant defect of cardiac conduction that is characterized by an abnormal ST-segment in leads V1-V3 on the ELECTROCARDIOGRAM resembling a right BUNDLE-BRANCH BLOCK; high risk of VENTRICULAR TACHYCARDIA; or VENTRICULAR FIBRILLATION; SYNCOPAL EPISODE; and possible sudden death. This syndrome is linked to mutations of gene encoding the cardiac SODIUM CHANNEL alpha subunit.
The thoracolumbar division of the autonomic nervous system. Sympathetic preganglionic fibers originate in neurons of the intermediolateral column of the spinal cord and project to the paravertebral and prevertebral ganglia, which in turn project to target organs. The sympathetic nervous system mediates the body's response to stressful situations, i.e., the fight or flight reactions. It often acts reciprocally to the parasympathetic system.
A short vein that collects about two thirds of the venous blood from the MYOCARDIUM and drains into the RIGHT ATRIUM. Coronary sinus, normally located between the LEFT ATRIUM and LEFT VENTRICLE on the posterior surface of the heart, can serve as an anatomical reference for cardiac procedures.
Modified cardiac muscle fibers composing the terminal portion of the heart conduction system.
AMINO ALCOHOLS containing the propanolamine (NH2CH2CHOHCH2) group and its derivatives.
A class I anti-arrhythmic agent (one that interferes directly with the depolarization of the cardiac membrane and thus serves as a membrane-stabilizing agent) with a depressant action on the heart similar to that of guanidine. It also possesses some anticholinergic and local anesthetic properties.
A general class of ortho-dihydroxyphenylalkylamines derived from tyrosine.
A tetrameric calcium release channel in the SARCOPLASMIC RETICULUM membrane of SMOOTH MUSCLE CELLS, acting oppositely to SARCOPLASMIC RETICULUM CALCIUM-TRANSPORTING ATPASES. It is important in skeletal and cardiac excitation-contraction coupling and studied by using RYANODINE. Abnormalities are implicated in CARDIAC ARRHYTHMIAS and MUSCULAR DISEASES.
The amount of BLOOD pumped out of the HEART per beat, not to be confused with cardiac output (volume/time). It is calculated as the difference between the end-diastolic volume and the end-systolic volume.
Symptoms of cerebral hypoperfusion or autonomic overaction which develop while the subject is standing, but are relieved on recumbency. Types of this include NEUROCARDIOGENIC SYNCOPE; POSTURAL ORTHOSTATIC TACHYCARDIA SYNDROME; and neurogenic ORTHOSTATIC HYPOTENSION. (From Noseworthy, JH., Neurological Therapeutics Principles and Practice, 2007, p2575-2576)
The dilatation of the aortic wall behind each of the cusps of the aortic valve.
In screening and diagnostic tests, the probability that a person with a positive test is a true positive (i.e., has the disease), is referred to as the predictive value of a positive test; whereas, the predictive value of a negative test is the probability that the person with a negative test does not have the disease. Predictive value is related to the sensitivity and specificity of the test.
A type of automatic, not reentrant, ectopic ventricular rhythm with episodes lasting from a few seconds to a minute which usually occurs in patients with acute myocardial infarction or with DIGITALIS toxicity. The ventricular rate is faster than normal but slower than tachycardia, with an upper limit of 100 -120 beats per minute. Suppressive therapy is rarely necessary.
Computer-assisted processing of electric, ultrasonic, or electronic signals to interpret function and activity.
Developmental abnormalities involving structures of the heart. These defects are present at birth but may be discovered later in life.
Production of an image when x-rays strike a fluorescent screen.
A nucleoside that is composed of ADENINE and D-RIBOSE. Adenosine or adenosine derivatives play many important biological roles in addition to being components of DNA and RNA. Adenosine itself is a neurotransmitter.
The ENTERIC NERVOUS SYSTEM; PARASYMPATHETIC NERVOUS SYSTEM; and SYMPATHETIC NERVOUS SYSTEM taken together. Generally speaking, the autonomic nervous system regulates the internal environment during both peaceful activity and physical or emotional stress. Autonomic activity is controlled and integrated by the CENTRAL NERVOUS SYSTEM, especially the HYPOTHALAMUS and the SOLITARY NUCLEUS, which receive information relayed from VISCERAL AFFERENTS.
The position or attitude of the body.
The muscular structure separating the right and the left lower chambers (HEART VENTRICLES) of the heart. The ventricular septum consists of a very small membranous portion just beneath the AORTIC VALVE, and a large thick muscular portion consisting of three sections including the inlet septum, the trabecular septum, and the outlet septum.
A condition in which the LEFT VENTRICLE of the heart was functionally impaired. This condition usually leads to HEART FAILURE; MYOCARDIAL INFARCTION; and other cardiovascular complications. Diagnosis is made by measuring the diminished ejection fraction and a depressed level of motility of the left ventricular wall.
The muscle tissue of the HEART. It is composed of striated, involuntary muscle cells (MYOCYTES, CARDIAC) connected to form the contractile pump to generate blood flow.
An alkaloid, originally from Atropa belladonna, but found in other plants, mainly SOLANACEAE. Hyoscyamine is the 3(S)-endo isomer of atropine.
A significant drop in BLOOD PRESSURE after assuming a standing position. Orthostatic hypotension is a finding, and defined as a 20-mm Hg decrease in systolic pressure or a 10-mm Hg decrease in diastolic pressure 3 minutes after the person has risen from supine to standing. Symptoms generally include DIZZINESS, blurred vision, and SYNCOPE.
Abrupt changes in the membrane potential that sweep along the CELL MEMBRANE of excitable cells in response to excitation stimuli.
Glycosides from plants of the genus DIGITALIS. Some of these are useful as cardiotonic and anti-arrhythmia agents. Included also are semi-synthetic derivatives of the naturally occurring glycosides. The term has sometimes been used more broadly to include all CARDIAC GLYCOSIDES, but here is restricted to those related to Digitalis.
A form of CARDIAC MUSCLE disease, characterized by left and/or right ventricular hypertrophy (HYPERTROPHY, LEFT VENTRICULAR; HYPERTROPHY, RIGHT VENTRICULAR), frequent asymmetrical involvement of the HEART SEPTUM, and normal or reduced left ventricular volume. Risk factors include HYPERTENSION; AORTIC STENOSIS; and gene MUTATION; (FAMILIAL HYPERTROPHIC CARDIOMYOPATHY).
Pathologic processes that affect patients after a surgical procedure. They may or may not be related to the disease for which the surgery was done, and they may or may not be direct results of the surgery.
A combination of congenital heart defects consisting of four key features including VENTRICULAR SEPTAL DEFECTS; PULMONARY STENOSIS; RIGHT VENTRICULAR HYPERTROPHY; and a dextro-positioned AORTA. In this condition, blood from both ventricles (oxygen-rich and oxygen-poor) is pumped into the body often causing CYANOSIS.
The hemodynamic and electrophysiological action of the HEART ATRIA.
A beta-1 adrenergic antagonist that has been used in the emergency treatment of CARDIAC ARRYTHMIAS.
A voltage-gated sodium channel subtype that mediates the sodium ion PERMEABILITY of CARDIOMYOCYTES. Defects in the SCN5A gene, which codes for the alpha subunit of this sodium channel, are associated with a variety of CARDIAC DISEASES that result from loss of sodium channel function.
This structure includes the thin muscular atrial septum between the two HEART ATRIA, and the thick muscular ventricular septum between the two HEART VENTRICLES.
A beta-2 adrenergic agonist used in the treatment of ASTHMA and BRONCHIAL SPASM.
A characteristic symptom complex.
Injections made into a vein for therapeutic or experimental purposes.
Transmission of the readings of instruments to a remote location by means of wires, radio waves, or other means. (McGraw-Hill Dictionary of Scientific and Technical Terms, 4th ed)
Irregular HEART RATE caused by abnormal function of the SINOATRIAL NODE. It is characterized by a greater than 10% change between the maximum and the minimum sinus cycle length or 120 milliseconds.
A disorder of cardiac function caused by insufficient blood flow to the muscle tissue of the heart. The decreased blood flow may be due to narrowing of the coronary arteries (CORONARY ARTERY DISEASE), to obstruction by a thrombus (CORONARY THROMBOSIS), or less commonly, to diffuse narrowing of arterioles and other small vessels within the heart. Severe interruption of the blood supply to the myocardial tissue may result in necrosis of cardiac muscle (MYOCARDIAL INFARCTION).
A cardioselective beta-1 adrenergic blocker possessing properties and potency similar to PROPRANOLOL, but without a negative inotropic effect.
An ethanolamine derivative that is an adrenergic alpha-1 agonist. It is used as a vasoconstrictor agent in the treatment of HYPOTENSION.
A form of inherited long QT syndrome (or LQT7) that is characterized by a triad of potassium-sensitive periodic paralysis, VENTRICULAR ECTOPIC BEATS, and abnormal features such as short stature, low-set ears, and SCOLIOSIS. It results from mutations of KCNJ2 gene which encodes a channel protein (INWARD RECTIFIER POTASSIUM CHANNELS) that regulates resting membrane potential.
A non-selective beta-adrenergic antagonist with a long half-life, used in cardiovascular disease to treat arrhythmias, angina pectoris, and hypertension. Nadolol is also used for MIGRAINE DISORDERS and for tremor.
Controlled physical activity which is performed in order to allow assessment of physiological functions, particularly cardiovascular and pulmonary, but also aerobic capacity. Maximal (most intense) exercise is usually required but submaximal exercise is also used.
The active sympathomimetic hormone from the ADRENAL MEDULLA. It stimulates both the alpha- and beta- adrenergic systems, causes systemic VASOCONSTRICTION and gastrointestinal relaxation, stimulates the HEART, and dilates BRONCHI and cerebral vessels. It is used in ASTHMA and CARDIAC FAILURE and to delay absorption of local ANESTHETICS.
Electric conductors through which electric currents enter or leave a medium, whether it be an electrolytic solution, solid, molten mass, gas, or vacuum.
Visualization of the heart structure and cardiac blood flow for diagnostic evaluation or to guide cardiac procedures via techniques including ENDOSCOPY (cardiac endoscopy, sometimes refered to as cardioscopy), RADIONUCLIDE IMAGING; MAGNETIC RESONANCE IMAGING; TOMOGRAPHY; or ULTRASONOGRAPHY.
The continuous measurement of physiological processes, blood pressure, heart rate, renal output, reflexes, respiration, etc., in a patient or experimental animal; includes pharmacologic monitoring, the measurement of administered drugs or their metabolites in the blood, tissues, or urine.
Failure of equipment to perform to standard. The failure may be due to defects or improper use.
The 10th cranial nerve. The vagus is a mixed nerve which contains somatic afferents (from skin in back of the ear and the external auditory meatus), visceral afferents (from the pharynx, larynx, thorax, and abdomen), parasympathetic efferents (to the thorax and abdomen), and efferents to striated muscle (of the larynx and pharynx).
Forced expiratory effort against a closed GLOTTIS.
Use of electric potential or currents to elicit biological responses.
Surgery performed on the heart.
Disorders of the AUTONOMIC NERVOUS SYSTEM occurring as a primary condition. Manifestations can involve any or all body systems but commonly affect the BLOOD PRESSURE and HEART RATE.
Patient care procedures performed during the operation that are ancillary to the actual surgery. It includes monitoring, fluid therapy, medication, transfusion, anesthesia, radiography, and laboratory tests.
A localized bulging or dilatation in the muscle wall of a heart (MYOCARDIUM), usually in the LEFT VENTRICLE. Blood-filled aneurysms are dangerous because they may burst. Fibrous aneurysms interfere with the heart function through the loss of contractility. True aneurysm is bound by the vessel wall or cardiac wall. False aneurysms are HEMATOMA caused by myocardial rupture.
A response by the BARORECEPTORS to increased BLOOD PRESSURE. Increased pressure stretches BLOOD VESSELS which activates the baroreceptors in the vessel walls. The net response of the CENTRAL NERVOUS SYSTEM is a reduction of central sympathetic outflow. This reduces blood pressure both by decreasing peripheral VASCULAR RESISTANCE and by lowering CARDIAC OUTPUT. Because the baroreceptors are tonically active, the baroreflex can compensate rapidly for both increases and decreases in blood pressure.
A condition caused by dysfunctions related to the SINOATRIAL NODE including impulse generation (CARDIAC SINUS ARREST) and impulse conduction (SINOATRIAL EXIT BLOCK). It is characterized by persistent BRADYCARDIA, chronic ATRIAL FIBRILLATION, and failure to resume sinus rhythm following CARDIOVERSION. This syndrome can be congenital or acquired, particularly after surgical correction for heart defects.
Naturally occurring or experimentally induced animal diseases with pathological processes sufficiently similar to those of human diseases. They are used as study models for human diseases.
Expenditure of energy during PHYSICAL ACTIVITY. Intensity of exertion may be measured by rate of OXYGEN CONSUMPTION; HEAT produced, or HEART RATE. Perceived exertion, a psychological measure of exertion, is included.
Abnormally low BLOOD PRESSURE that can result in inadequate blood flow to the brain and other vital organs. Common symptom is DIZZINESS but greater negative impacts on the body occur when there is prolonged depravation of oxygen and nutrients.
The HEART and the BLOOD VESSELS by which BLOOD is pumped and circulated through the body.
A prediction of the probable outcome of a disease based on a individual's condition and the usual course of the disease as seen in similar situations.
Pathological conditions involving the HEART including its structural and functional abnormalities.
The hemodynamic and electrophysiological action of the left HEART VENTRICLE. Its measurement is an important aspect of the clinical evaluation of patients with heart disease to determine the effects of the disease on cardiac performance.
A local anesthetic and cardiac depressant used as an antiarrhythmia agent. Its actions are more intense and its effects more prolonged than those of PROCAINE but its duration of action is shorter than that of BUPIVACAINE or PRILOCAINE.
An infant during the first month after birth.
Binary classification measures to assess test results. Sensitivity or recall rate is the proportion of true positives. Specificity is the probability of correctly determining the absence of a condition. (From Last, Dictionary of Epidemiology, 2d ed)
Theoretical representations that simulate the behavior or activity of the cardiovascular system, processes, or phenomena; includes the use of mathematical equations, computers and other electronic equipment.
A condition in which the RIGHT VENTRICLE of the heart was functionally impaired. This condition usually leads to HEART FAILURE or MYOCARDIAL INFARCTION, and other cardiovascular complications. Diagnosis is made by measuring the diminished ejection fraction and a depressed level of motility of the right ventricular wall.
An imbalance between myocardial functional requirements and the capacity of the CORONARY VESSELS to supply sufficient blood flow. It is a form of MYOCARDIAL ISCHEMIA (insufficient blood supply to the heart muscle) caused by a decreased capacity of the coronary vessels.
The heart rate of the FETUS. The normal range at term is between 120 and 160 beats per minute.
Contractile activity of the MYOCARDIUM.
A paravertebral sympathetic ganglion formed by the fusion of the inferior cervical and first thoracic ganglia.
Drugs that selectively bind to and activate beta-adrenergic receptors.
The hemodynamic and electrophysiological action of the HEART VENTRICLES.
Striated muscle cells found in the heart. They are derived from cardiac myoblasts (MYOBLASTS, CARDIAC).
The relationship between the dose of an administered drug and the response of the organism to the drug.
A heterogeneous condition in which the heart is unable to pump out sufficient blood to meet the metabolic need of the body. Heart failure can be caused by structural defects, functional abnormalities (VENTRICULAR DYSFUNCTION), or a sudden overload beyond its capacity. Chronic heart failure is more common than acute heart failure which results from sudden insult to cardiac function, such as MYOCARDIAL INFARCTION.
An imprecise term which may refer to a sense of spatial disorientation, motion of the environment, or lightheadedness.
Electromagnetic waves with frequencies between about 3 kilohertz (very low frequency - VLF) and 300,000 megahertz (extremely high frequency - EHF). They are used in television and radio broadcasting, land and satellite communications systems, radionavigation, radiolocation, and DIATHERMY. The highest frequency radio waves are MICROWAVES.
Precursor of epinephrine that is secreted by the adrenal medulla and is a widespread central and autonomic neurotransmitter. Norepinephrine is the principal transmitter of most postganglionic sympathetic fibers and of the diffuse projection system in the brain arising from the locus ceruleus. It is also found in plants and is used pharmacologically as a sympathomimetic.
An aspect of personal behavior or lifestyle, environmental exposure, or inborn or inherited characteristic, which, on the basis of epidemiologic evidence, is known to be associated with a health-related condition considered important to prevent.
Serotonin derivative proposed as potentiator for hypnotics and sedatives.
Agents that have a strengthening effect on the heart or that can increase cardiac output. They may be CARDIAC GLYCOSIDES; SYMPATHOMIMETICS; or other drugs. They are used after MYOCARDIAL INFARCT; CARDIAC SURGICAL PROCEDURES; in SHOCK; or in congestive heart failure (HEART FAILURE).
The artificial substitution of heart and lung action as indicated for HEART ARREST resulting from electric shock, DROWNING, respiratory arrest, or other causes. The two major components of cardiopulmonary resuscitation are artificial ventilation (RESPIRATION, ARTIFICIAL) and closed-chest CARDIAC MASSAGE.
A nicotinic antagonist most commonly used as an experimental tool. It has been used as a ganglionic blocker in the treatment of hypertension but has largely been supplanted for that purpose by more specific drugs.
The qualitative or quantitative estimation of the likelihood of adverse effects that may result from exposure to specified health hazards or from the absence of beneficial influences. (Last, Dictionary of Epidemiology, 1988)
A selective adrenergic beta-1 blocking agent that is commonly used to treat ANGINA PECTORIS; HYPERTENSION; and CARDIAC ARRHYTHMIAS.
The long-term (minutes to hours) administration of a fluid into the vein through venipuncture, either by letting the fluid flow by gravity or by pumping it.
Diseases of the parasympathetic or sympathetic divisions of the AUTONOMIC NERVOUS SYSTEM; which has components located in the CENTRAL NERVOUS SYSTEM and PERIPHERAL NERVOUS SYSTEM. Autonomic dysfunction may be associated with HYPOTHALAMIC DISEASES; BRAIN STEM disorders; SPINAL CORD DISEASES; and PERIPHERAL NERVOUS SYSTEM DISEASES. Manifestations include impairments of vegetative functions including the maintenance of BLOOD PRESSURE; HEART RATE; pupil function; SWEATING; REPRODUCTIVE AND URINARY PHYSIOLOGY; and DIGESTION.
The process of generating three-dimensional images by electronic, photographic, or other methods. For example, three-dimensional images can be generated by assembling multiple tomographic images with the aid of a computer, while photographic 3-D images (HOLOGRAPHY) can be made by exposing film to the interference pattern created when two laser light sources shine on an object.
An abnormally low volume of blood circulating through the body. It may result in hypovolemic shock (see SHOCK).
A beta-adrenergic antagonist used in the treatment of hypertension, angina pectoris, arrhythmias, and anxiety.
Any operation on the spinal cord. (Stedman, 26th ed)
A state characterized by loss of feeling or sensation. This depression of nerve function is usually the result of pharmacologic action and is induced to allow performance of surgery or other painful procedures.
Nerves and plexuses of the autonomic nervous system. The central nervous system structures which regulate the autonomic nervous system are not included.
Drugs that bind to and block the activation of ADRENERGIC BETA-1 RECEPTORS.
Post-systolic relaxation of the HEART, especially the HEART VENTRICLES.
Studies to determine the advantages or disadvantages, practicability, or capability of accomplishing a projected plan, study, or project.
A guanidinium antihypertensive agent that acts by blocking adrenergic transmission. The precise mode of action is not clear.
Optical imaging techniques used for recording patterns of electrical activity in tissues by monitoring transmembrane potentials via FLUORESCENCE imaging with voltage-sensitive fluorescent dyes.
Drugs that mimic the effects of stimulating postganglionic adrenergic sympathetic nerves. Included here are drugs that directly stimulate adrenergic receptors and drugs that act indirectly by provoking the release of adrenergic transmitters.
The measurement of magnetic fields generated by electric currents from the heart. The measurement of these fields provides information which is complementary to that provided by ELECTROCARDIOGRAPHY.
A statistical technique that isolates and assesses the contributions of categorical independent variables to variation in the mean of a continuous dependent variable.
A congenital heart defect characterized by downward or apical displacement of the TRICUSPID VALVE, usually with the septal and posterior leaflets being attached to the wall of the RIGHT VENTRICLE. It is characterized by a huge RIGHT ATRIUM and a small and less effective right ventricle.

Site of myocardial infarction. A determinant of the cardiovascular changes induced in the cat by coronary occlusion. (1/1171)

The influence of site of acute myocardial infarction on heart rate, blood pressure, cardiac output, total peripheral resistance (TPR), cardiac rhythm, and mortality was determined in 58 anesthetized cats by occlusion of either the left anterior descending (LAD), left circumflex or right coronary artery. LAD occlusion resulted in immediate decrease in cardiac output, heart rate, and blood pressure, an increase in TPR, and cardiac rhythm changes including premature ventricular beats, ventricular tachycardia, and occasionally ventricular fibrillation. The decrease in cardiac output and increase in TPR persisted in the cats surviving a ventricular arrhythmia. In contrast, right coronary occlusion resulted in a considerably smaller decrease in cardiac output. TPR did not increase, atrioventricular condition disturbances were common, and sinus bradycardia and hypotension persisted in the cats recovering from an arrhythmia. Left circumflex ligation resulted in cardiovascular changes intermediate between those produced by occlusion of the LAD or the right coronary artery. Mortality was similar in each of the three groups. We studied the coronary artery anatomy in 12 cats. In 10, the blood supply to the sinus node was from the right coronary artery and in 2, from the left circumflex coronary artery. The atrioventricular node artery arose from the right in 9 cats, and from the left circumflex in 3. The right coronary artery was dominant in 9 cats and the left in 3. In conclusion, the site of experimental coronary occlusion in cats is a major determinant of the hemodynamic and cardiac rhythm changes occurring after acute myocardial infarction. The cardiovascular responses evoked by ligation are related in part to the anatomical distribution of the occluded artery.  (+info)

Regional differences in the recovery course of tachycardia-induced changes of atrial electrophysiological properties. (2/1171)

BACKGROUND: Regional differences in recovery of tachycardia-induced changes of atrial electrophysiological properties have not been well studied. METHODS AND RESULTS: In the control group (5 dogs), atrial effective refractory period (AERP) and inducibility of atrial fibrillation (AF) were assessed before and every 4 hours for 48 hours after complete atrioventricular junction (AVJ) ablation with 8-week VVI pacing. In experimental group 1 (15 dogs), AERP and inducibility of AF were assessed before and after complete AVJ ablation with 8-week rapid right atrial (RA) pacing (780 bpm) and VVI pacing. In experimental group 2 (7 dogs), AERP and inducibility of AF were assessed before and after 8-week rapid left atrial (LA) pacing and VVI pacing. AERP and inducibility and duration of AF were obtained from 7 epicardial sites. In the control group, atrial electrophysiological properties obtained immediately and during 48-hour measurements after pacing did not show any change. In the 2 experimental groups, recovery of atrial electrophysiological properties included a progressive recovery of AERP shortening, recovery of AERP maladaptation, and decrease of duration and episodes of reinduced AF. However, recovery of shortening and maladaptation of AERP and inducibility of AF was slower at the LA than at the RA and Bachmann's bundle. CONCLUSIONS: The LA had a slower recovery of tachycardia-induced changes of atrial electrophysiological properties, and this might play a critical role in initiation of AF.  (+info)

Ketotifen and cardiovascular effects of xamoterol following single and chronic dosing in healthy volunteers. (3/1171)

AIMS: To study whether desensitization occurs after long-term administration of the 1-adrenoceptor partial agonist xamoterol and, if so, whether this can be influenced by ketotifen. METHODS: In a double-blind, randomized design 10 young, healthy males received ketotifen (2 x 1 mg day(-1) p.o.) or placebo for 3 weeks with xamoterol (2 x 200 mg day(-1) p.o.) administered concomitantly during the last 2 weeks. 'l1-adrenoceptor mediated responses were assessed as exercise-induced tachycardia and isoprenaline-induced shortening of heart rate corrected electromechanical systole (QS2c); isoprenaline-induced tachycardia was measured as a mixed beta1-/beta2-adrenoceptor-mediated effect. RESULTS: The first dose of xamoterol significantly increased resting heart rate and systolic blood pressure and significantly shortened QS2c. The last dose of xamoterol after 2 weeks of treatment still produced the same responses. Ketotifen did not influence these effects of xamoterol on resting haemodynamics. The first dose of xamoterol caused a rightward shift of the exercise- and isoprenaline-induced tachycardia (mean dose ratios+/-s.e.mean: 1.20+/-0.05 and 2.46+/-0.23) and the isoprenaline-evoked shortening of QS2c (dose ratio 3.59+/-0.68). This rightward shift was even more pronounced after 2 weeks xamoterol treatment. This additional rightward shift after 2 weeks of xamoterol was not affected by ketotifen (mean difference (95% CI) of log transformed dose ratios between placebo and ketotifen: exercise tachycardia 0.001 (-0.03; 0.04); isoprenaline tachycardia 0.03 (-0.15; 0.21); isoprenaline induced shortening of QS2c 0.13 (-0.22; 0.48)). CONCLUSIONS: In humans xamoterol is a partial beta1-adrenoceptor agonist with positive chrono- and inotropic effects at rest and antagonistic properties under conditions of beta-adrenoceptor stimulation. These effects were well maintained after chronic dosing with no signs of beta1-adrenoceptor desensitization. Ketotifen does not change the beta-adrenoceptor mediated responses of xamoterol after chronic dosing.  (+info)

LocaLisa: new technique for real-time 3-dimensional localization of regular intracardiac electrodes. (4/1171)

BACKGROUND: Estimation of the 3-dimensional (3D) position of ablation electrodes from fluoroscopic images is inadequate if a systematic lesion pattern is required in the treatment of complex arrhythmogenic substrates. METHODS AND RESULTS: We developed a new technique for online 3D localization of intracardiac electrodes. Regular catheter electrodes are used as sensors for a high-frequency transthoracic electrical field, which is applied via standard skin electrodes. We investigated localization accuracy within the right atrium, right ventricle, and left ventricle by comparing measured and true interelectrode distances of a decapolar catheter. Long-term stability was analyzed by localization of the most proximal His bundle before and after slow pathway ablation. Electrogram recordings were unaffected by the applied electrical field. Localization data from 3 catheter positions, widely distributed within the right atrium, right ventricle, or left ventricle, were analyzed in 10 patients per group. The relationship between measured and true electrode positions was highly linear, with an average correlation coefficient of 0.996, 0.997, and 0.999 for the right atrium, right ventricle, and left ventricle, respectively. Localization accuracy was better than 2 mm, with an additional scaling error of 8% to 14%. After 2 hours, localization of the proximal His bundle was reproducible within 1.4+/-1.1 mm. CONCLUSIONS: This new technique enables accurate and reproducible real-time localization of electrode positions in cardiac mapping and ablation procedures. Its application does not distort the quality of electrograms and can be applied to any electrode catheter.  (+info)

Effects of pacing-induced and balloon coronary occlusion ischemia on left atrial function in patients with coronary artery disease. (5/1171)

OBJECTIVES: The aim of this study was to compare left atrial (LA) function in 16 patients with distal left anterior descending (LAD) and in 16 patients with proximal left circumflex (LCx) coronary artery stenosis at rest and immediately after pacing-induced tachycardia (LAD-pacing [P] and LCx-P) or coronary occlusion (LAD-CO and LCx-CO). BACKGROUND: During left ventricular (LV) ischemia, compensatory augmentation of LA contraction enhances LV filling and performance. The left atrium is supplied predominantly by branches arising from the LCx. Therefore, we hypothesized that one mechanism for the loss of atrial contraction may be ischemic LA dysfunction. METHODS: Left ventricular and LA pressure-area relations were derived from simultaneous double-tip micromanometer pressure recordings and automatic boundary detection echocardiograms. RESULTS: Immediately after pacing or after coronary occlusion, LV end-diastolic pressure, LV relaxation, LA mean pressure and LV stiffness significantly increased in all patients. However, the area of the A loop of the LA pressure-area relation, representing the LA pump function, significantly decreased in groups LCx-P and LCx-CO (from 14+/-3 to 9+/-2, and from 16+/-4 to 9+/-2 mm Hg.cm2, respectively, p < 0.05), whereas it increased in groups LAD-P and LAD-CO (from 12+/-3 to 54+/-10, and from 16+/-3 to 49+/-8 mm Hg.cm2, respectively, p < 0.001). CONCLUSIONS: In patients with LAD stenosis, LV supply or demand ischemia is associated with enhanced LA pump function. However, in patients with proximal LCx stenosis who develop the same type and degree of ischemia, LA branches might have been affected, rendering the LA ischemic and unable to increase its booster pump function.  (+info)

Frequency of arrhythmias and other cardiac abnormalities in fulminant hepatic failure. (6/1171)

In a series of 106 patients with fulminant hepatic failure and grade 4 encephalopathy, cardiac arrhythmias and other abnormalities occurred in 92 per cent. The most common was sinus tachycardia (75%) and this was the only abnormality in 22 per cent of the patients. Sudden cardiac arrest occurred in 25 per cent, various ectopic beats in 20 per cent, and heart block or bradycardia in 18 per cent. Other electrocardiographic abnormalities, mostly of the T wave and ST segment, were found in 31 per cent. Cardiac and respiratory arrests were usually unrelated to each other and both frequently occurred without warning. Only 7 out of 71 patients with arrhythmias other than sinus tachycardia survived, compared with 15 out of 31 patients without them (P less than 0-005). During the latter part of the series when an arrhythmia computer was used to monitor 38 patients, it was shown that significantly lower arterial oxygen levels occurred in those with arrhythmias, other than sinus tachycardia, than in those without. They were also found to be more acidotic and hyperkalaemic, and a higher number required dialysis and ventilation. Macroscopical cardiac abnormalities including scattered petechial haemorrhages, small pericardial effusions, and fatty, pale, and flabby ventricles, were found at necropsy in 64 per cent of the patients examined. Combinations of these macroscopical abnormalities occurred, particularly in the paracetamol overdose group. Another necropsy finding of possible significance in the pathogenesis of arrhythmias was cerebral oedema, present in 48 per cent of the patients examined, and often associated with coning of the brain stem. However, 7 of the 16 patients who suffered asystolic cardiac arrests had no macroscopical abnormality of either heart or brain. In the management of patients with fulminant hepatic failure continuous cardiac monitoring is essential. Correction of the biochemical and coagulation defects may decrease the frequency of arrhythmias but studies of the mechanism and control of cerebral oedema and its relation to cardiovascular function are urgently needed.  (+info)

Sino-aortic denervation augments the increase in blood pressure seen during paradoxical sleep in the rat. (7/1171)

Using a computer assisted telemetric system, we have re-examined the effect of sino-aortic denervation (SAD) on the changes in arterial blood pressure (AP) and heart rate (HR) during sleep in the rat suitably recovered from the operation. Eight 1 hourly polygraphic recordings were performed 4 weeks after the initial SAD surgery. In the SAD rats, the increase in AP during paradoxical sleep (PS) was much larger than that in sham-operated rats. HR in the SAD rats increased on-going from slow-wave sleep to PS, but it showed no change in sham-operated rats. The present study suggests that chronic SAD causes the enhanced AP increase during PS concomitantly with the persistent hypertension and tachycardia across sleep-wake states.  (+info)

Aryl propanolamines: comparison of activity at human beta3 receptors, rat beta3 receptors and rat atrial receptors mediating tachycardia. (8/1171)

1. The in vitro activity of four aryl propanolamines was compared to two prototypic beta3 receptor agonists, CGP 12177 and CL316243 at the human beta3 receptor, the rat beta3 receptor in the stomach fundus and receptors mediating atrial tachycardia. 2. L-739,574 was the most potent (EC50 = 9 nM) and selective agonist at the human beta3 receptor with high maximal response (74% of the maximal response to isoproterenol). 3. A phenol-biaryl ether analogue possessed modest affinity for the human beta3 receptor (EC50 = 246 nM), but was highly efficacious with a maximal response 82% of the maximal response to isoproterenol. The other derivatives were intermediate in potency with low maximal responses. 4. These agonists at the human beta3 receptor did not activate the rat beta3 receptor in the rat stomach fundus. In fact, the aryl propanolamines (10(-6) M) inhibited CL316243-induced activation of the rat beta3 receptor. Thus, agonist activity at the human beta3 receptor translated into antagonist activity at the rat beta3 receptor. 5. L739,574 and the phenol biaryl ether increased heart rate via beta1 receptors. 6. Although CGP12177 produced atrial tachycardia, neither the indole sulphonamide nor biphenyl biaryl ether did, although both had high affinity for the human beta3 receptor. Thus, the atrial tachycardic receptor was not identical to the human beta3 receptor. 7. These studies (a) characterized four aryl propanolamines with high affinity at the human beta3 receptor, (b) found that they were antagonists at the rat beta3 receptor, an observation with profound implications for in vivo rat data, and (c) established that the rodent atrial non-beta1, beta2 or beta3 tachycardic receptor was also unrelated to the human beta3 receptor.  (+info)

Tachycardia, also called tachyarrhythmia, is a heart rate that exceeds the normal resting rate. In general, a resting heart rate over 100 beats per minute is accepted as tachycardia in adults. Heart rates above the resting rate may be normal (such as with exercise) or abnormal (such as with electrical problems within the heart). The upper threshold of a normal human resting heart rate is based on age. Cutoff values for tachycardia in different age groups are fairly well standardized; typical cutoffs are listed below: 1-2 days: Tachycardia > 159 beats per minute (bpm) 3-6 days: Tachycardia >166 bpm 1-3 weeks: Tachycardia >182 bpm 1-2 months: Tachycardia >179 bpm 3-5 months: Tachycardia >186 bpm 6-11 months: Tachycardia >169 bpm 1-2 years: Tachycardia >151 bpm 3-4 years: Tachycardia >137 bpm 5-7 years: Tachycardia >133 bpm 8-11 years: Tachycardia >130 bpm 12-15 years: Tachycardia >119 bpm >15 years - adult: Tachycardia >100 bpm Heart rate is considered in the context of the prevailing clinical ...
Mechanistic studies of human heart failure are complicated by the prolonged time course of development of the disease, the technical challenges of isolating cardiac tissue or cells from explanted hearts, the inability to investigate the early time course of cellular alterations, and the lack of control over experimental conditions. Thus, it is fortunate that the canine tachycardia-induced heart failure model so closely reproduces the known hemodynamic and ionic changes that have been identified in human hearts. The present findings indicate that, in addition to the electrophysiological changes noted in earlier studies, significant alterations in Ca2+ handling occur in isolated myocytes from failing hearts, following the general pattern of human studies. Through biochemical and functional measurements in the same hearts, we have found strong evidence in support of the hypothesis that the induction of heart failure triggers a shift in the balance of cytosolic Ca2+ extrusion mechanisms from SR Ca2+ ...
Successful Ablation for Atrial Tachycardia Originated from Sinus Venosa with Tachycardia-Induced Cardiomyopathy. . Biblioteca virtual para leer y descargar libros, documentos, trabajos y tesis universitarias en PDF. Material universiario, documentación y tareas realizadas por universitarios en nuestra biblioteca. Para descargar gratis y para leer online.
Fetal Tachycardia (FT) is described as increase in baseline fetal heart rate (FHR) above 160bpm. Mild fetal tachycardia is described as 161-180bpm and severe tachycardia is defined as greater than 180bpm for at least three minutes. The fetal tachycardia causes include maternal fever, dehydration or anxiety, maternal ketosis, medications like anticholinergic medications, sympathomimetic medications like terbutaline, fetal movement, preterm fetus, maternal thyrotoxicosis and maternal anaemia1. Fetal tachycardia is considered significant (any range ,160-180bpm) in the presence of maternal pyrexia as Chorioamnionitis is suspected. Fetal arrhythmia or congenital defect is associated with FHR more than 200 bpm. Baseline FHR tachycardia represents an increase in sympathetic and or a decrease in parasympathetic autonomic nervous system tone1.. Complicated fetal tachycardia in the presence of decelerations or maternal fever qualify the decision for delivering the baby in view of fetal distress and ...
Tachycardia refers to a condition of an irregularly rapid beating of the heart. Tachycardia is defined as a heart rate greater than 100 beats per minute, while a normal heart beats is measured at 72 beats per minute. Tachycardia occurs when the electrical impulses that coordinate heartbeats do not function properly and cause the heart to beat irregularly fast. In some situations, an increased heart rate can be normal, such as during exercise and stress. However, depending on the extent of the Tachycardia and the overall health of the patient, tachycardia may be dangerous and require serious medical attention.. If the heart rate is abnormally increased for an extended period of time, oxygen and carbon dioxide levels in the bloodstream can be dramatically altered. Tachycardia can cause stress on the heart because the heart muscle is forced to take less time to relax between each contraction. Patients with Tachycardia may often feel out of breath because the faster the heart beats, the more oxygen ...
TY - JOUR. T1 - Tachycardia in Post-Infarction Hearts. T2 - Insights from 3D Image-Based Ventricular Models. AU - Arevalo, Hermenegild. AU - Plank, Gernot. AU - Helm, Patrick. AU - Halperin, Henry R. AU - Trayanova, Natalia. PY - 2013/7/2. Y1 - 2013/7/2. N2 - Ventricular tachycardia, a life-threatening regular and repetitive fast heart rhythm, frequently occurs in the setting of myocardial infarction. Recently, the peri-infarct zones surrounding the necrotic scar (termed gray zones) have been shown to correlate with ventricular tachycardia inducibility. However, it remains unknown how the latter is determined by gray zone distribution and size. The goal of this study is to examine how tachycardia circuits are maintained in the infarcted heart and to explore the relationship between the tachycardia organizing centers and the infarct gray zone size and degree of heterogeneity. To achieve the goals of the study, we employ a sophisticated high-resolution electrophysiological model of the infarcted ...
A pacing system provided with a mode switching feature and ventricular rate regularization (VRR) function adapted to stabilize or regularize ventricular heart rate during chronic or paroxysmal atrial tachyarrhythmia. In a preferred embodiment, the pacing system nominally operates in an atrial synchronized pacing mode such as DDD or DDDR pacing mode. In response to detection of atrial rhythm characteristics consistent with an atrial tachyarrhythmia, e.g., atrial fibrillation, a mode switch into a non-atrial synchronized, ventricular rate regularization pacing mode, e.g. DDIR or VDIR pacing mode, is made. If the VRR function is programmed on, the ventricular pacing rate based upon a rate responsive sensor derived ventricular pacing rate modulated on a beat by beat basis by preceding intrinsic or paced ventricular events, the stability of the intrinsic ventricular heart rate, and any atrial pace events to regularize the ventricular pacing rate. The pacing system may also be permanently programmed to the
The freeMD virtual doctor has found 32 conditions that can cause Abdominal Tenderness and Tachycardia. There are 6 common conditions that can cause Abdominal Tenderness and Tachycardia. There are 4 somewhat common conditions that can cause Abdominal Tenderness and Tachycardia. There are 7 uncommon conditions that can cause Abdominal Tenderness and Tachycardia. There are 15 rare conditions that can cause Abdominal Tenderness and Tachycardia.
This page includes the following topics and synonyms: Tachycardia due to Medications, Drug Induced Tachycardia, Toxin Induced Tachycardia.
The study of conversion of AF to sinus rhythm provides information on changes associated with the resumption of atrial contraction and AV synchrony leading to a regularized, rate-controlled ventricular response. Previous studies have documented improvement in cardiac output ([75]) and cardiopulmonary exercise testing variables of ventricular rate control, maximal oxygen uptake and anaerobic threshold ([76, 77]) after cardioversion to sinus rhythm. More recently, a small, prospective study of patients with a diagnosis of idiopathic dilated cardiomyopathy and chronic AF reported a significant increase in left ventricular ejection fraction after pharmacologic or electrical cardioversion to sinus rhythm ([78]). Whether improvement in cardiac function was due to restoration of atrial systolic function and AV synchrony versus reversal of an underlying cardiomyopathy associated with rapid and irregular ventricular rates during AF was not resolved by this study. Subsequent study of atrial and ...
The best 16 synonyms for tachycardia, including: hypotension, tachypnoea, dysrhythmias, bronchospasm, palpitation, dyspnoea, stridor, low blood pressure, hypoxaemia, ventricular-fibrillation, oedema and more... Find another word for tachycardia at YourDictionary.
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The rhythm is a regular WCT ( = Wide-Complex Tachycardia) at ~ 210/minute without sign of atrial activity. Dr. Smith has reviewed the principal differential diagnosis. I dont think QRS duration is particularly helpful here, given that the patient is a 12-year old child (for whom normal QRS duration is a bit less than in adults). So the QRS is definitely wide, though not excessively so - but probably overlaps most entities to be considered. What strikes me - is the obvious suggestion of bifascicular block (RBBB/LAHB), though of atypical morphology. In an otherwise healthy child (who does not have congenital heart disease or other structural heart abnormality) - one would expect a more typical RBBB morphology with a clear triphasic appearance with taller right-rabbit ear in lead V1, instead of the atypical picture we see here in V1,V2,V3. Considering this and integrating it with the important Learning Points Dr. Smith emphasizes (ie, that fascicular VT often occurs in otherwise normal hearts) - ...
Most of us (assuming no pediatric cardiologists are listening) do not see a large number of pediatric arrhythmias (certainly far less than weve all seen in adults). As a result - level of comfort in both diagnosis and management tends to be less than that attained with adult arrhythmias. As wonderfully emphasized by Dr. Smith by the 2 pediatric arrhythmia examples shown here - accurate determination of rate is CRITICAL in the assessment of pediatric arrhythmias. For this determination - I have found use of the Every-other-Beat (or sometimes, Every-third-Beat) method a user-friendly way to rapidly and accurate determine heart rate for any regular fast rhythm. While most of us recall, 300- 150 - 100 - 75 - 60 - 50 as the answers for rate calculation when the R-R interval of a regular tachycardia is 1, 2, 3, 4, 5 or 6 large boxes, respectively - this becomes problematic with very fast rates. Thats because small differences in measurements may translate into significant under- or ...
Improved methods and devices perform tachycardia detection and anti-tachycardia pacing (ATP) to convert a tachycardia (e.g., VT or AT) to normal sinus rhythm. According to one embodiment, an anti-tachycardia pacing method includes sensing, during sinus rhythm, first and second cardiac signals at first and second sites, respectively, in a patients heart. The first and second sites include left and right ventricles or left and right atria, for example. The method further includes sensing third and fourth cardiac signals at the first and second sites, respectively, during a tachycardia (e.g., ventricular tachycardia or atrial tachycardia). The cardiac signals are processed to provide respective values. One or more anti-tachycardia pacing pulses are delivered at the site closest to the reentrant circuit based on a comparison of a first ratio of the first and third values and a second ratio of the second and fourth values. Unipolar sensing of the cardiac signals may be employed by, for example,
Tachycardia: Abnormally rapid heartbeat, usually with a HEART RATE above 100 beats per minute for adults. Tachycardia accompanied by disturbance in the cardiac depolarization (cardiac arrhythmia) is called tachyarrhythmia.
TITLE PAGETitle: Interesting changeover from short VA to long VA tachycardia with single atrial premature depolarisation: What is the mechanism?Short title: Concomitant AVNRT and atrial tachycardia: Killing two birds with one stone.
What is tachycardia arrhythmia? Learn about tachycardia arrhythmia (fast heartbeat), including causes, risk factors, symptoms, diagnosis and treatment from the cardiology experts at Mercy Health.
Fascinating case with a number of interesting insights to learn. Diagnosis of alternate preexcitation on the last tracing is subtle, but important to recognize. The 2 other thoughts I considered in my differential diagnosis for this last ECG were: i) electrical alternans; and ii) ventricular bigeminy with fusion from end-diastolic PVCs - but neither fit (thats because we see DELTA waves in several leads on this tracing that provide the diagnosis). Otherwise - the T wave peaking we see on the tachycardia ECGs is extremely interesting. I have often seen T wave peaking with tachycardia (and on stress tests) in otherwise healthy young adults - but I dont expect ST elevation. In addition, this patient was apparently markedly dehydrated on each occasion when the ECG was done. Hyperkalemia (perhaps secondary to volume contraction) not uncommonly leads to T wave peaking with recurrent episodes, but again supposedly not associated with ST elevation (Wonder what serum K+ values were in this patient ...
Tachycardia refers to a fast resting heart rate, usually over 100 beats per minute. Tachycardia can be dangerous, depending on its underlying cause and on
I. Problem/Condition. Tachycardias include all heart rhythms with a rate , 100 bpm. They can be divided into two primary categories: ) Narrow Complex Tachycardias (NCT) which have a QRS duration , 120 msec ) Wide Complex Tachycardias (WCT) which have a QRS duration , 120 msec The NCTs are due to rapid activation of…. ...
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BACKGROUND: Catheter ablation (CA) therapy is the first choice treatment in adults with heart rhythm disturbances. The arrhythmias in adults are mainly conditioned by coronary disease. Etiology of arrhythmias in children is mostly associated with inherited heart disorders. According to the current guidelines the CA is widely used in children, indicating the need to make it more achievable in pediatric population. AIM: To assess efficacy and safety of CA in children with different types of arrhythmias on the initial learning curve at newly built Ablation Center in The Independent Pediatric Hospital of Medical University of Warsaw ...
Most arrhythmia including Tachycardia are not serious However, any abnormality associated with the heart can quickly develop into a serious, or even life
An interesting technicality in the use of the PERC rule to rule out pulmonary embolism is the tachycardia component - it asks not whether the patient is tachycardic at the time of the application of the rule, or whether tachycardia was sustained throughout the emergency department stay, but instead whether the patient had (as described by Jeff Kline in his great review article on PE diagnosis and risk stratification): 3. Pulse ,100 beats/min during entire stay in ED. Meaning, even transient tachycardia may suggest a life-threatening diagnosis, even if it resolves while the patient is in the emergency department, and were probably PERCing out a whole bunch of patients inappropriately, at least according to Kline (who, notably, testifies a whole bunch as an expert witness in cases of missed pulmonary emboli).. I recently had a handful of patients in whom concerning blood pressures were measured and documented, which then resolved when vital signs were re-checked or after a small quantity of ...
You are working one evening in the emergency department when a 60-something year old female is slotted for a room. Her chief complaint? Fever, weakness, vomiting. Seeing that her triage heart rate was 157, you leave your granola bar where it is and immediately walk into the room to assess her. You see an elderly-appearing female in moderate respiratory distress. Her temperature is 38.2, blood pressure is 125/87, RR is 32, oxygen saturation is 93% on 5L NC. She has a history of a bone-marrow transplant and is chronically immunosuppressed. She endorses poor PO intake and several episodes of emesis over the last few days. She says that she came in today when she developed some shortness of breath as well. She denies any chest pain or palpitations. On exam, her mucous membranes are dry, her abdomen non-tender, and her breath sounds are decreased in the right base. You are a bit disturbed by the looks of her rhythm strip on the monitor so you get a 12-lead EKG ...
A Computational Study of the Effects of Tachycardia-Induced Remodeling on Calcium Wave Propagation in Rabbit Atrial Myocytes: In atrial cardiomyocytes without a
Learn more about Tachycardia at Reston Hospital Center DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...
Learn more about Tachycardia at Grand Strand Medical Center DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...
Tachycardia is a condition where a persons heart is beating abnormally fast. When we become more active our heart rate increases to meet the increased
Atrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics (costs for program #250319) ✔ University Hospital Halle (Saale) ✔ Department of Cardiology and Angiology (Department of Internal Medicine III) ✔ BookingHealth.com
Atrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics (costs for program #144277) ✔ Academic Hospital Neuperlach ✔ Department of Cardiology, Pneumology and Internal Intensive Care ✔ BookingHealth.com
Tachycardia is a rapid heart rate caused by a problem in the hearts electrical system. Find out more about this common heart rhythm disorder (arrhythmia).
A pacemaker-mediated tachycardia (PMT) can be defined as any condition in which a pacemaker paces the ventricles at rates that are inappropriately fast. This can be due to (1) a rate response setting that is too sensitive, (2) tracking of atrial noise (such as what may occur with electromagnetic interference), (3) inappropriate pacemaker mani...
A heart rate of more than 100 beats per minute (BPM) in adults is called tachycardia. Whats too fast for you may depend on your age and physical condition.
Rapid and abnormal increase in heart rate more than 100 beats a minute in rest is tachycardia. It is caused by abnormal electrical impulses originated in the heart chambers with a physiological or pathological background.
Its been a while since I been on here, but I must say life is good, I am truly blessed. My pvcs are almost completely gone, I may feel only a couple a week and I havent had a tachycardia attack in over three months, I must say again God is good. I was once afraid to live on my own because if my heart issues, but I recently moved into my own house three weeks ago. I posted a while back about the doctor putting me on Mirtazapine for anxiety and weight gain, it has really helped me alot, I started off at 111lbs, I am now 134lbs ...
Question - Suffering from SVT tachycardia. Prescribed with metroprolol. Is it safe to go for skiing?. Ask a Doctor about diagnosis, treatment and medication for Increased heart rate, Ask a Cardiologist
how high can an axiety attact drive ones HR up? to 110, 120, 130?? I fhigh, one will confuse the adrenaline caused high HR that stops in a few minutes with tachycardia. More and more I believe, aft...
i did a post on here regarding tachycardia warnings well this is part 2 after suffering complications in hospital which resulted in very low potassium it would seem that i am back to square one...
Dont just change employers, change your life with Healthworks, Inc. This webinar focuses on the core concepts of a basic electrophysiology study and tools to induce tachycardia.
A disruption in the normal electrical impulses controlling the pumping action of the heart causes tachycardia, according to Mayo Clinic. Many different factors cause these disruptions and the related...
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Had cathetor ablation following a 189 heart beat which I could not control (prior to that over a number of years had, say 120 beats - I could stop by holding my breath a few times and it would disappe
I have been experiencing a fast, pounding heart rate for over a month now. It gets worse when I stand up. I get light headed, very short of breath, dizzy, weak, and feel as if Im going to faint. I wen...
I have been wearing a chest hr monitor for the last week and have been concerned that I seem to be tachycardiac in the afternoons. My resting heart...
DI-fusion, le Dépôt institutionnel numérique de lULB, est loutil de référencementde la production scientifique de lULB.Linterface de recherche DI-fusion permet de consulter les publications des chercheurs de lULB et les thèses qui y ont été défendues.
TY - JOUR. T1 - Chronic orthostatic intolerance. T2 - A disorder with discordant cardiac and vascular sympathetic control. AU - Furlan, Raffaello. AU - Jacob, Giris. AU - Snell, Marie. AU - Robertson, David. AU - Porta, Alberto. AU - Harris, Paul. AU - Mosqueda-Garcia, Rogelio. PY - 1998/11/17. Y1 - 1998/11/17. N2 - Background - Chronic orthostatic intolerance (COI) is a debilitating autonomic condition in young adults. Its neurohumoral and hemodynamic profiles suggest possible alterations of postural sympathetic function and of baroreflex control of heart rate (HR). Methods and Results - In 16 COI patients and 16 healthy volunteers, intra-arterial blood pressure (BP), ECG, central venous pressure (CVP), and muscle sympathetic nerve activity (MSNA) were recorded at rest and during 75°tilt. Spectral analysis of RR interval and systolic arterial pressure (SAP) variabilities provided indices of sympathovagal modulation of the sinoatrial node (ratio of low-frequency to high-frequency components, ...
Objectives. The purpose of this study was to determine the predictors of electrically induced ventricular tachycardia in a large sample of patients with unexplained syncope and to examine the value of the signal-averaged electrocardiogram (ECG) in those patient subsets with varying pretest probability of ventricular tachycardia.. Background. In patients with unexplained syncope, electrophysiologic study can provide important diagnostic information, such as inducibility of ventricular tachycardia. The signal-averaged ECG can predict inducible ventricular tachycardia, but its utility has not been prospectively studied in a large group of patients with unexplained syncope.. Methods. At six hospitals, 189 consecutive patients with unexplained syncope underwent signal-averaged ECG and electrophysiologic studies.. Results. Ventricular tachycardia was induced in 28 patients (15%). Univariate predictors of ventricular tachycardia included history of previous myocardial infarction, reduced left ...
There are several ways to classify ventricular tachycardia, such as morphology or based on the duration of the episodes. From a morphological point of view, ventricular tachycardia can be divided into monomorphic ventricular tachycardia, scar related monomorphic ventricular tachycardia, polymorphic ventricular tachycardia and RVOT tachycardia. The second type of tachycardia mentioned here is the most common, especially with patients who have already suffered from a heart attack and it can also lead to death. Based on the duration, ventricular tachycardia can be divided into two types: non-sustained ventricular tachycardia if the fast rhythm ends itself within thirty seconds and sustained ventricular tachycardia if the rhythm lasts longer than thirty seconds.. Determining what causes is can be very complicated and requires the input of a specialized doctor and several tests, the most common of which is effectuated with the help of an EKG. The manifestations of ventricular tachycardia can be very ...
This condition can cause dizziness when moving to an upright position and can be triggered by infections. They found that older people, women, and those who had five or more symptoms in the first week of becoming ill with Covid-19 were more likely to develop long Covid. Researchers have analysed data from the COVID Symptom Study app to discover who is most at risk of developing long Covid. The BHF is supporting the following research projects: The BHF is supporting the following research projects: The BHF is supporting the following research projects: The most common symptoms are feeling lightheaded, palpitations (being aware of your heartbeat) and fatigue. The most common symptoms are feeling lightheaded, palpitations (being aware of your heartbeat) and fatigue. There is emerging evidence that a few people who develop long Covid have similar symptoms to people with a condition known as postural tachycardia syndrome, or postural orthostatic tachycardia syndrome (PoTS).. ...
The Postural Tachycardia Syndrome (POTS) is the most common disorder seen in autonomic clinics. Cardinal hemodynamic feature of this chronic and debilitating disorder of orthostatic tolerance is an exaggerated orthostatic tachycardia (≥30 bpm increase in HR with standing) in the absence of orthostatic hypotension. There are multiple pathophysiological mechanisms that underlie POTS. Some patients with POTS have evidence of elevated sympathoneural tone. This hyperadrenergic state is likely a driver of the excessive orthostatic tachycardia. Another common pathophysiological mechanism in POTS is a hypovolemic state. Many POTS patients with a hypovolemic state have been found to have a perturbed renin-angiotensin-aldosterone profile. These include inappropriately low plasma renin activity and aldosterone levels with resultant inadequate renal sodium retention. Some POTS patients have also been found to have elevated plasma angiotensin II (Ang-II) levels, with some studies suggesting problems with
Radiofrequency ablation of ventricular tachycardia is now well established in the management of patients with focal ventricular tachycardia occurring without structural heart disease.1 ,2 ,11 Although high success rates have been reported in this context and much has been learnt about mapping and ablation, they form a small proportion of the total population of patients with ventricular tachycardia.8 ,9 Ischaemic heart disease is the dominant cause of ventricular tachycardia, and RF ablation in this setting is beset with problems. The traditional approach has been to induce ventricular tachycardia and then map the induced rhythm and perform ablation during the tachycardia, seeking to terminate it.. The conventional approach to mapping arrhythmias has been to identify the earliest ventricular activation, which is assumed to be at, or close, to the ventricular tachycardia origin. This approach works well in focal ventricular tachycardia or at surgery where a large endocardial resection is ...
Wide QRS tachycardia may be due to ventricular tachycardia (VT), supraventricular tachycardia (SVT) with aberrant conduction, or atrioventricular reentrant tachycardia (AVRT) with an accessory pathway. Children with wide QRS complex tachycardia may p
Introduction: Postural tachycardia syndrome (POTS) is a disorder of orthostatic intolerance characterized by excessive tachycardia of unknown etiology. Our objective in this study was to evaluate the correlation between C-fiber involvement as shown by skin biopsy and adrenergic cardiac metaiodobenzylguanadine (MIBG) uptake in POTS patients. Methods: Skin biopsies of 84 patients with POTS were examined by Protein Gene Product 9.5 (PGP9.5) immunohistochemistry and were compared with MIBG myocardial scintigraphy imaging data. Results: Mean intraepidermal nerve fiber (IENF) density was in the lower normal age-adjusted range, 7.2 ± 2.9/mm (normal ≥7/mm), and was slightly below the normal range in 45% of POTS patients. MIBG uptake was reduced in 21% of patients. Low IENF density correlated with reduced cardiac MIBG uptake (r = 0.39, P = 0.001). Conclusions: A subset of neuropathic POTS patients may harbor mild small fiber neuropathy with abnormalities of unmyelinated nerve fibers in the skin ...
Atrial tachycardia is a type of heart rhythm problem in which the hearts electrical impulse comes from an ectopic pacemaker (that is, an abnormally located cardiac pacemaker) in the upper chambers (atria) of the heart, rather than from the sinoatrial node, the normal origin of the hearts electrical activity. Atrial tachycardias can exhibit very regular (consistent) heart rates ranging typically from 140 to 220 beats per minute.[1] As with any other form of tachycardia (rapid heart beat), the underlying mechanism can be either the rapid discharge of an abnormal focus, the presence of a ring of cardiac tissue that gives rise to a circle movement (reentry),[2] or a triggered rapid rhythm due to other pathological circumstances (as would be the case with some drug toxicities, such as digoxin toxicity). Atrial tachycardia is a risk factor for atrial fibrillation, as the rapid rhythm can trigger or degrade into the lack of a rhythm.[3] All atrial tachycardias are by definition supraventricular ...
Providing information and support about postural orthostatic tachycardia syndrome (PoTS) for sufferers, medical professionals, family, and friends - aiming to raise awareness.
Providing information and support about postural orthostatic tachycardia syndrome (PoTS) for sufferers, medical professionals, family, and friends - aiming to raise awareness.
The morphology of the tachycardia depends on its cause and the origin of the re-entry electrical circuit in the heart. In monomorphic ventricular tachycardia, the shape of each heart beat on the ECG looks the same because the impulse is either being generated from increased automaticity of a single point in either the left or the right ventricle, or due to a reentry circuit within the ventricle. The most common cause of monomorphic ventricular tachycardia is scarring of the heart muscle from a previous myocardial infarction (heart attack). This scar cannot conduct electrical activity, so there is a potential circuit around the scar that results in the tachycardia. This is similar to the re-entrant circuits that are the cause of atrial flutter and the re-entrant forms of supraventricular tachycardia. Other rarer congenital causes of monomorphic VT include right ventricular dysplasia, and right and left ventricular outflow tract VT. Polymorphic ventricular tachycardia, on the other hand, is most ...
Objectives: Patients with postural tachycardia syndrome (POTS) often appear anxious and report inattention. Patients with POTS were formally assessed for psychiatric disorders and inattention and compared with patients with attention deficit hyperactivity disorder (ADHD) and control subjects.. Methods: Patients with POTS (n = 21), ADHD (n = 18) and normal control subjects (n = 20) were assessed for DSM-IV psychiatric disorders and completed a battery of questionnaires that assessed depression, anxiety and ADHD characteristics.. Results: Patients with POTS did not have an increased prevalence of major depression or anxiety disorders, including panic disorder, compared with the general population. Patients with POTS had mild depression. They scored as moderately anxious on the Beck Anxiety Inventory but did not exhibit a high level of anxiety sensitivity. Patients with POTS scored significantly higher on inattention and ADHD subscales than control subjects. These symptoms were not present during ...
What is ventricular tachycardia? Ventricular tachycardia is an arrhythmia represented by rapid heartbeats that originate in the ventricles. During ventricular tachycardia the heart may beat inefficiently and as a result the blood pressure may decrease very much. This type of tachycardia may lead to life-threatening arrhythmias and cardiac arrest. Treatment options for ventricular tachycardias include medications, catheter ablation or placement of an implantable cardioverter-defibrillator (ICD).
Background: When noninvasive studies remain negative, the diagnosis of unexplained tachycardia in the youth is a dilemma. Invasive electrophysiological study (EPS) requires generally anesthesia and hospitalization in young children. The purpose of the study was to determine the factors of negativity of transesophageal EPS in children and teenagers complaining of tachycardia.. Population: 267 children, aged from 6 to 19 years, mean 15±3, complained of tachycardia and had a normal ECG in sinus rhythm.. Methods: Transesophageal EPS was performed in out-patient clinic without sedation but in presence of parents; EPS consisted of atrial pacing and atrial stimulation with 1 and 2 extrastimuli in control state and after isoproterenol. Long-term Holter monitoring was systematic in patients with negative EPS.. Results: Supraventricular tachycardia (SVT)(AV node re-entrant tachycardia, AV re-entrant tachycardia, atrial tachycardia) was induced in 146 patients (group I) and EPS remained negative in 121 ...
Ventricular tachycardia is the term for a fast heart rate that begins in the ventricles, the lower section of the heart. It leads to a fast yet regular heart rhythm. However, it can lead to ventricular fibrillation, which is a fast yet irregular heart beat. With this, the heart may beat so quickly and irregularly that it actually stops pumping blood. This can lead to sudden cardiac arrest and death. However, with treatment ventricular tachycardia can be handled and wont develop into ventricular fibrillation.. At times, especially when it develops in children and teens, it can be difficult if not impossible to identify the cause of ventricular tachycardia. However, in most cases, some sort of heart disease is the base cause. This may include a congenital heart defect, a previous heart attack, myocarditis, or dilated or hypertrophic cardiomyopathy. At times, ventricular tachycardia develops after a person has heart surgery. Some medications can also cause ventricular tachycardia. These include ...
Introduction. Neonatal tachycardia is defined as a resting heart rate (HR) of 182 beats per minute (bpm) when the baby is not crying.1,2 Supraventricular tachycardia (SVT) is the most common tachyarrhythmia in the neonatal period and usually presents with a HR of more than 200bpm and with a narrow QRS complex. In 80% of patients, it originates through a mechanism of retrograde conduction via an accessory pathway between the ventricle and the atrium, with an abrupt onset and termination. In 15% of patients it results from atrial ectopic beats, while 5% of cases correspond to re-entrant nodal tachycardias.3,4 Approximately half of these patients present with heart failure, with suspicion of sepsis in some cases. The tachycardia may be detected in the foetal period, most frequently between weeks 28 and 33 of gestation, possibly manifesting with hydrops.5. The key element in the management of haemodynamically unstable patients with tachycardia is electric cardioversion. However, in stable patients, ...
TY - JOUR. T1 - Use of the impella™ microaxial blood pump for ablation of hemodynamically unstable ventricular tachycardia. AU - Abuissa, Hussam. AU - Roshan, John. AU - Lim, Bernard. AU - Asirvatham, Ssamuel J.. PY - 2010/4. Y1 - 2010/4. N2 - Impella™ for VT Ablation. Ablation for ventricular tachycardia remains a challenge with suboptimal procedural success rates. One of the major causes of difficulty is precipitous hypotension when ventricular tachycardia is induced precluding even rapid mapping of the arrhythmia. We report the successful use of the Impella™ microcirculatory axial blood flow pump in 3 patients with hemodynamically unstable ventricular tachycardia that allowed successful completion of the procedure. In these 3 patients, there was no evidence of Impella™-related valvular disturbance, iatrogenic ventricular arrhythmias, or interference with mapping and ablation catheter movement. (J Cardiovasc Electrophysiol, Vol. 21, pp. 458-4, April 2010). AB - Impella™ for VT ...
TY - JOUR. T1 - Optimal surface electrocardiogram lead for identification of the mechanism of supraventricular tachycardia in children. AU - Liberman, Leonardo. AU - Pass, Robert H.. AU - Starc, Thomas J.. PY - 2008/1/1. Y1 - 2008/1/1. N2 - OBJECTIVE: Although supraventricular tachycardia (SVT) can be identified from any lead of the bedside monitor, the mechanism of tachycardia is not always obvious. We analyzed the 12-lead electrocardiogram (ECG) in SVT of pediatric patients with different mechanisms of SVT to determine if there is a consistent optimal lead for rhythm identification. METHODS: Twelve-lead ECGs during SVT were available for retrospective analysis in 54 patients. The tachycardia mechanism was determined either by intracardiac or transesophageal recording, or after cardioversion analysis of atrial flutter or fibrillation. Blinded analysis of each separate lead of the 12-lead ECG was done to determine the best lead to diagnose the mechanism of tachycardia. For statistical analysis, ...
A closed-heart method for treating ventricular tachycardia in a myocardial infarct patient afflicted with ventricular tachycardia is disclosed. The method comprises, first, defining a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) of the patient, and then ablating the thin layer of spared myocardial tissue by a closed-heart procedure with an ablation catheter. Apparatus for carrying out the method is also disclosed, Also disclosed is a method for prognosing the likelihood of ventricular tachycardia occuring in a myocardial infarct patient not previously diagnosed as afflicted with ventricular tachycardia. The method comprises detecting a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) in the patient.
A closed-heart method for treating ventricular tachycardia in a myocardial infarct patient afflicted with ventricular tachycardia is disclosed. The method comprises, first, defining a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) of the patient, and then ablating the thin layer of spared myocardial tissue by a closed-heart procedure with an ablation catheter. Apparatus for carrying out the method is also disclosed. Also disclosed is a method for prognosing the likelihood of ventricular tachycardia occuring in a myocardial infarct patient not previously diagnosed as afflicted with ventricular tachycardia. The method comprises detecting a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) in the patient.
STARS PO Box 175 Stratford-upon-Avon Warwickshire CV37 8YD +44 (0) 1789 450 564 [email protected] www.stars.org.uk Postural Tachycardia Syndrome (PoTS) This information sheet is designed for patients who have been diagnosed with postural tachycardia syndrome (PoTS). It contains information on the diagnosis, treatment and management of PoTS. Postural tachycardia syndrome (PoTS) is an abnormality of functioning of the autonomic (involuntary) nervous system. It is defined as an increase in heart rate of over 30 beats per minute (or to higher than 120 beats per minute) (40 bpm in those age 12-19) when standing upright. Typically there is no postural fall in blood pressure, although fainting (syncope) can occur (see below). The symptoms can vary (see below) but mostly involve orthostatic intolerance (the inability to withstand the upright posture) and are often made worse by various daily activities, e.g., modest physical exertion or food. It is a disorder which is slowly being recognised by the ...
Re-entrant atrial tachycardia is usually seen after cardiac surgery or catheter ablation with linear lesions that result in islets of scars. In patients with structurally normal hearts, atrial tachycardia is associated with a low mortality rate. Objectives: Ann Intern Med. My cardiologist now tells me this is common in about 50% of patients and is most likely a temporary issue. It is usually a self-limiting condition, but can cause serious haemodynamic deterioration in the immediate postoperative phase .JET is a narrow QRS complex tachycardia, usually with atrioventricular dissociation and slower atrial than ventricular rate. JAMA. I recently (August 2018) had my 1st ablation at TCA in Austin TX. 2017;92(1):98-105. Arrhythmias are common after cardiac surgery such as coronary artery bypass grafting surgery. Dual-loop circuits in postoperative atrial macro re-entrant tachycardias. 2007;89:91-5. reported that the most common site of focal ATs after AF ablation was the previously isolated PV. ...
Ventricular tachycardia is a very rare fetal arrhythmia accounting for fewer than 2% of fetal tachycardias. We describe a fetus presenting at 30 weeks gestation with ventricular tachycardia at a rate of 220 beats per min and fetal hydrops. The tachycardia was unresponsive to flecainide but was co...
Looking for Sinus tachycardia? Find out information about Sinus tachycardia. see arrhythmia arrhythmia , disturbance in the rate or rhythm of the heartbeat. Various arrhythmias can be symptoms of serious heart disorders; however,... Explanation of Sinus tachycardia
Looking for online definition of auricular tachycardia in the Medical Dictionary? auricular tachycardia explanation free. What is auricular tachycardia? Meaning of auricular tachycardia medical term. What does auricular tachycardia mean?
The heart has its own electrical system that controls the heart rate and contraction of the heart, which pumps blood throughout the body. Normal heart rates vary based on a persons age.. A rapid heart rate can be caused by many different factors. Sometimes tachycardia is the bodys response to a trigger like fever, anxiety, exercise, or pain, just to name a few. It also can be due to an abnormal rhythm in the heart (called a dysrhythmia), such as supraventricular tachycardia (SVT).. Congenital heart abnormalities, high blood pressure, medications, dehydration, caffeine, smoking, and recreational drug use also can cause tachycardia. Tachycardias can lead to dizziness, shortness of breath, chest pain, fainting, and heart palpitations (a sensation of feeling the heart beat fast or irregularly). Some tachycardias are life threatening and can lead to blood clots, causing a stroke, heart failure, and sudden death.. ...
The subjects are patients with COI and adult healthy volunteers of similar age, gender, and body mass.. Participation in this protocol is offered to people 18 years old or older, independently of gender, race, age, ethnicity, religion, or any other demographic or sociopolitical classifications.. The subjects in the second off-site study are family members of the identified proband. Participation in this protocol is offered to people 18 years old or older, independently of gender, race, ethnicity, religion, or any other demographic or sociopolitical classifications.. In the second off-site study, participation is also offered to subjects 10 to 17 years old, provided that appropriate informed consent/assent has been given by the subject and his/her parent or guardian.. EXCLUSION CRITERIA:. Age: Minors younger than 18 years old are excluded, except for the off-site study. Advanced age does not constitute an exclusion criterion. For subjects more than 55 years old, carotid Doppler studies are done, ...
This is an irregular narrow complex rhythm. The first 2 sinus P waves (S) are of the same morphology with a PRI of about 0.20 sec and a rate of about 60 bpm (lead II). The tachycardia starts with a premature complex with different P wave morphology (blue arrow). The tachycardia is best observed in V1. The rate tends to increase (107 to about 170 bpm) then there is a non-conducted P wave (first red arrow). The first conducted P has a shorter PRI compared to the previous PRI. The tachycardia resumes with 1:1 conduction then a non-conducted P wave is seen (second red arrow). Thus, this is atrial tachycardia with a block (type I block). ...
View details of top chronic orthostatic hypotension hospitals in Bangalore. Get guidance from medical experts to select best chronic orthostatic hypotension hospital in Bangalore
Researchers discover autoantibodies in POTS patients that may account for increased circulation of norepinephrine and tachycardia upon standing.
TY - JOUR. T1 - Mode of induction of ventricular tachycardia and prognosis in patients with coronary disease. T2 - The multicenter unsustained tachycardia trial (MUSTT). AU - Piccini, Jonathan P.. AU - Hafley, Gail E.. AU - Lee, Kerry L.. AU - Fisher, John D.. AU - Josephson, Mark E.. AU - Prystowsky, Eric N.. AU - Buxton, Alfred E.. PY - 2009/8/1. Y1 - 2009/8/1. N2 - Mode of Induction in MUSTT. Introduction: Programmed stimulation is an important prognostic tool in the evaluation of patients with an ejection fraction ≤40% after myocardial infarction. Many believe that ventricular tachycardia (VT) requiring 3 ventricular extrastimuli (VES) for induction is less likely to occur spontaneously and has less predictive value. However, it is unknown whether the mode of VT induction is associated with long-term prognosis. Methods and Results: We analyzed a cohort of 371 patients enrolled in MUSTT who had inducible monomorphic VT and who were not treated with antiarrhythmic drugs or an implantable ...
POTS is a relaytively common cause of disabilty in young people, characterized by symptoms associated with fast heartbeat (tachycardia) upon standing.
With monomorphic ventricular tachycardia all of the QRS waves will likely be symmetrical. Every ventricular impulse is being created from the identical place in the ventricles thus all of the QRS
Case report A 31 year old female was admitted to A/E 6 hours after ingestion of 90 bupropion tablets (13.5g), with alcohol. Initially, the patient was conscious but disorientated, GCS 12/15, agitated, tachycardia (124 beats per minute), raised blood pressure (180/96), tremor, dystonia, increased muscle tone with brisk tendon reflexes and clonus. Activated charcoal was administered but the patient had a convulsion and aspirated. She deteriorated further with recurrent seizures, GCS 5/15, BP 120/60 and heart rate 120 beats per minute and required intubation, ventilation and sedation with diazepam. The ECG showed broad complex tachycardia, which reverted to normal sinus rhythm after treatment with adenosine 12mg iv (Figure 1). The patient was ventilated for 2 days during which she remained hypotensive and had extrapyramidal signs. She was extubated after 3 days. Discussion Bupropion, an atypical antidepressant is structurally similar to amphetamine and diethylpropion.2 It was originally approved ...
Dive into the research topics of Outer loop and isthmus in ventricular tachycardia circuits: Characteristics and implications. Together they form a unique fingerprint. ...
During wide complex tachycardia (heart rate , 100/min, QRS , 0.12 sec) the differentiation between supraventricular and ventricular origin of the arrhythmia is important to guide therapy. Several algorithms have been developed to aid in this differentiation (below). It is important to keep in mind that a good estimate of VT versus SVT can be made based on the clinical vignette: ...
Tachycardia induced cardiomyopathy is a potentially lethal cause of heart failure generally because of atrial tachycardia and less frequently ventricular tachycardia. We present two cases of Marines with severe traumatic blast injuries secondary to i
There are 2 main new findings in the present study. The first one is that, after removing autonomic influences, inhibition of endogenous NO synthesis with L-NMMA results in significant bradycardia. This effect is not explained by its pressor effect or baroreflex responses, as the same increase in SBP with phenylephrine did not reduce HR during autonomic blockade. These data show that NO has a tonic effect on human HR in vivo, and that this effect is independent of the autonomic control of HR. It is well known that NO is produced within the heart not only by endothelial cells, but also by myocytes.20,21 It has been proposed that NO has a role in the regulation of cardiac function both under normal and pathological conditions.11,22,23 NOS inhibition was previously shown to reduce HR in patients with transplanted, and presumably denervated, hearts.24 Our findings indicate for the first time that endogenous NO tonically regulates HR in healthy subjects. This is also in agreement with studies in ...
Symptomatic falls in blood pressure after standing or eating are a frequent clinical problem, particularly in the elderly. The symptoms are often due to cerebral hypoperfusion and include generalized weakness, dizziness or lightheadedness, visual blu
The heartbeat is controlled by the electrical system of the heart. This system is made up of several parts that tell the muscle of the heart when to contract. The SA node starts the heartbeat, causing the atria, or upper chambers of the heart, to contract. The signal then travels through the AV node, bundle of His, bundle branches, and Purkinje fibers. This causes the ventricles, the lower chambers of the heart, to contract. This organized flow of electrical signals produces a normal heartbeat. Normal heartbeats can be seen in an Electrocardiogram or ECG.. Focal Ventricular Tachycardia is a heart arrhythmia, or irregular heartbeat. During focal ventricular tachycardia, accelerated abnormal electrical pulses in the lower chambers, or ventricles, disrupt the normal firing of the SA node, causing the heart to beat rapidly. The abnormal signals originate in a specific area in the ventricles called a focus. This irregular heartbeat can be seen on an electrocardiogram.. A rapid heartbeat does not give ...
Ventricular tachycardia (VT) is a type of regular, fast heart rate that arises from improper electrical activity in the ventricles of the heart.. ​. Ventricular tachycardia may last for only a few seconds, or it can last for much longer. You may feel dizzy or short of breath, or have chest pain.. Although a few seconds may not result in problems, longer periods are dangerous.. ​. Sometimes, ventricular tachycardia can cause your heart to stop (cardiac arrest), which is a life-threatening medical emergency.. ​. Many different conditions can cause ventricular tachycardia. Its important to get a prompt, accurate diagnosis and appropriate care.. ​. The best way to prevent ventricular tachycardia is to treat or eliminate risk factors that may lead to heart disease. If you already have heart disease, follow your treatment plan and a heart-healthy lifestyle.. See your doctor if you have any problems with the heartbeat. In some cases, urgent care is needed. ...
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Ventricular Tachycardia Treatment in Frisco - Ventricular Tachycardia is the issue related to the heart. If we talk about Texas, many hospitals provide the be
... >169 bpm 1-2 years: Tachycardia >151 bpm 3-4 years: Tachycardia >137 bpm 5-7 years: Tachycardia >133 bpm 8-11 years ... Tachycardia >159 beats per minute (bpm) 3-6 days: Tachycardia >166 bpm 1-3 weeks: Tachycardia >182 bpm 1-2 months: Tachycardia ... AV nodal reentrant tachycardia (AVNRT) is the most common reentrant tachycardia. It is a regular narrow complex tachycardia ... flutter AV nodal reentrant tachycardia Accessory pathway mediated tachycardia Atrial tachycardia Multifocal atrial tachycardia ...
Most automatic tachycardias are supraventricular tachycardias (SVT). It is important to recognise an automatic tachycardia ... is an automatic tachycardia. Atrial fibrillation may be considered an automatic tachycardia. Junctional ectopic tachycardia, in ... These tachycardias, or fast heart rhythms, differ from reentrant tachycardias (AVRT and AVNRT) in which there is an abnormal ... Idioventricular tachycardia is notable because it is the only automatic tachycardia which is not an SVT. Treatment depends on ...
Thus, sinus tachycardia is a medical finding that can be either physiological or pathological. Tachycardia is often ... Treatment for physiologic sinus tachycardia involves treating the underlying causes of the tachycardia response. Beta blockers ... In adults, sinus tachycardia is defined as a heart rate greater than 100 beats per minute (bpm). The normal resting heart rate ... Sinus tachycardia can present in more than a third of the patients with AMI but this usually decreases over time. Patients with ...
Forms of junctional tachycardia include junctional ectopic tachycardia (JET) and atrioventricular nodal re-entrant tachycardia ... Junctional tachycardia is a form of supraventricular tachycardia characterized by involvement of the AV node. It can be ... It is a tachycardia associated with the generation of impulses in a focus in the region of the atrioventricular node due to an ... "Differentiating atrioventricular nodal reentrant tachycardia from junctional tachycardia: novel application of the delta H-A ...
A Tachycardia is the medical term as in Supraventricular tachycardia. Tachycardia may also refer to: Tachycardia: A Journal, an ... online book by Satsvarupa dasa Goswami This disambiguation page lists articles associated with the title Tachycardia. If an ...
... tachycardia that is seen. Junctional ectopic tachycardia (JET) is a rare tachycardia caused by increased automaticity of the AV ... AV nodal reentrant tachycardia (AVNRT) or junctional reciprocating tachycardia (JRT) AV reciprocating tachycardia (AVRT) - ... Multifocal atrial tachycardia (MAT) is tachycardia arising from at least three ectopic foci within the atria, distinguished by ... Supraventricular Tachycardia Sound Sound of a 20 year old male's heart in an episode of paroxysmal supraventricular tachycardia ...
... is a form of tachycardia which begins and ends in an acute (or paroxysmal) manner. It is also known as " ... It can be divided by the origin: supraventricular tachycardia ventricular tachycardia synd/3076 at Who Named It? L. Bouveret. ... Making sense of the ECG 3rd edition p 119 "paroxysmal tachycardia" at Dorland's Medical Dictionary v t e (Articles with short ...
Forms of atrial tachycardia (ATach) include multifocal atrial tachycardia (MAT), focal atrial tachycardia and atrial flutter. ... Paroxysmal atrial tachycardia (PAT) is an episode of arrhythmia that begins and ends abruptly. Atrial tachycardia tends to ... of patients presenting for supraventricular tachycardia (SVT) ablation had atrial tachycardia. Electrocardiographic features ... Atrial tachycardia is a type of heart rhythm problem in which the heart's electrical impulse comes from an ectopic pacemaker ( ...
B Garner, J; M Miller, J (April 2013). "Wide Complex Tachycardia - Ventricular Tachycardia or Not Ventricular Tachycardia, That ... Ventricular tachycardia may turn into ventricular fibrillation and can result in cardiac arrest. Ventricular tachycardia can ... It may be very difficult to differentiate between ventricular tachycardia and a wide-complex supraventricular tachycardia in ... Right ventricular outflow tract (RVOT) tachycardia is a type of monomorphic ventricular tachycardia originating in the right ...
... this is called multifocal atrial tachycardia (if the heart rate is ≤100, this is technically not a tachycardia and it is then ... Bradley DJ, Fischbach PS, Law IH, Serwer GA, Dick M (August 2001). "The clinical course of multifocal atrial tachycardia in ... Then, if the heart rate exceeds 100 beats per minute, the phenomenon is called multifocal atrial tachycardia.[citation needed] ... In select cases of refractory multifocal atrial tachycardia, AV node ablation has been performed. Studies have found an average ...
Supraventricular tachycardia Sinus tachycardia Postural orthostatic tachycardia syndrome Dysautonomia Metabolic myopathies ... Inappropriate sinus tachycardia (IST) is a rare type of cardiac arrhythmia within the category of supraventricular tachycardia ... "Postural Tachycardia Syndrome and Inappropriate Sinus Tachycardia: Role of Autonomic Modulation and Sinus Node Automaticity". ... Exclusion of all other causes of sinus tachycardia Common forms of supraventricular tachycardia (SVT) must be excluded Normal P ...
"Supraventricular Tachycardia, Junctional Ectopic Tachycardia: Overview - eMedicine". Retrieved 21 December 2008. Campbell, R. W ... Junctional ectopic tachycardia (JET) is a rare syndrome of the heart that manifests in patients recovering from heart surgery. ... Tachycardia (from the Greek takhys, meaning "swift", and kardia, meaning heart) means a swift heart rate. By this definition, ... Junctional ectopic tachycardia derives its name from the problem it causes. "Junctional" is used as the abnormal tissue driving ...
... (TIC) is a disease where prolonged tachycardia (a fast heart rate) or arrhythmia (an ... TIC has been associated with supraventricular tachycardia (SVT), ventricular tachycardia (VT), frequent premature ventricular ... atrioventricular reciprocating tachycardia, and atrioventricular nodal reentry tachycardia. Atrial fibrillation is the most ... Treatment of TIC can involve treating the heart failure as well as the tachycardia or arrhythmia. TIC has a good prognosis with ...
... (PSVT) is a type of supraventricular tachycardia, named for its intermittent episodes ... AV nodal re-entrant tachycardia (AVNRT) makes up 56% of cases Atrioventricular reentrant tachycardia (AVRT) makes up 27% of ... "Tachycardia , Fast Heart Rate". American Heart Association. Archived from the original on 12 April 2013. Retrieved 19 April ... About 2.3 per 1000 people have paroxysmal supraventricular tachycardia. Problems typically begin in those 12 to 45 years old. ...
... (AVRT), or atrioventricular reciprocating tachycardia, is a type of abnormal fast heart ... Between episodes of tachycardia the affected person is likely to be asymptomatic; however, the ECG would demonstrate the ... AV nodal reentrant tachycardia Electrical conduction system of the heart Wolff-Parkinson-White syndrome Permanent junctional ... Adult tachycardia algorithm. 2010. (Articles with short description, Short description matches Wikidata, All articles with ...
September 2012). "Desmopressin acutely decreases tachycardia and improves symptoms in the postural tachycardia syndrome". Heart ... Postural tachycardia syndrome was coined in 1982 in a description of a patient who had postural tachycardia, but not ... and tachycardia. People with POTS can be misdiagnosed with inappropriate sinus tachycardia (IST) as they present similarly. One ... "Propranolol decreases tachycardia and improves symptoms in the postural tachycardia syndrome: less is more". Circulation. 120 ( ...
The ventricular tachycardia may take a characteristic form known as bidirectional ventricular tachycardia. This form of ... The bidirectional ventricular tachycardia associated with this condition was described in 1975. The term "Catecholaminergic ... Catecholaminergic polymorphic ventricular tachycardia (CPVT) is an inherited genetic disorder that predisposes those affected ... In those with CPVT, catecholamine release can lead to an abnormal heart rhythm or arrhythmia known as ventricular tachycardia. ...
AV-nodal reentrant tachycardia (AVNRT) is a type of abnormal fast heart rhythm. It is a type of supraventricular tachycardia ( ... AV nodal reentrant tachycardia is the most common regular supraventricular tachycardia. It is more common in women than men ( ... "Atrioventricular Nodal Reentrant Tachycardia (AVNRT)". 2009-09-30. Shen, Sharon; Knight, Bradley P. (2014), Kibos, Ambrose S.; ... Rosero, Spencer (2015), "A Brief Overview of Supraventricular Tachycardias", in Huang, MD, David T.; Prinzi, MD, Travis (eds ...
... (PJRT) is a rare cardiac arrhythmia. It is a supraventricular tachycardia, and a ... "Tachycardia-mediated cardiomyopathy and the permanent form of junctional reciprocating tachycardia." Texas Heart Institute ... This means that the associated tachycardia may be subclinical and only diagnosed at a late stage, after significant damage to ... Without treatment, the prevalence of tachycardia-induced cardiomyopathy has been reported to be between 20% and 50%, however ...
A tachycardia-dependent bundle branch block is a defect in the conduction system of the heart, and is distinct from typical ... Tachycardia-dependent bundle branch block (TDBBB) can affect either ventricle in the heart, and occurs when the heart's rate of ... Tachycardia-dependent bundle branch block can prevent both ventricles from contracting efficiently and can limit the cardiac ... For example, certain situations of excessive or reduced heart rate (tachycardia or bradycardia, respectively) can cause a BBB ...
"Tachycardia. Start of July 2008 - Part 10". GN Press. Retrieved 7 March 2009. "Radhadesh Newsletter" (PDF). ISKCON Radhadesh. ...
"Tachycardia". www.sdgonline.org. Retrieved August 7, 2008. Goswami, Satsvarupa dasa. "India Journal 2008". www.sdgonline.org. " ...
... tachycardia; bizarre, and possibly violent behavior; and severe mydriasis with resultant painful photophobia that can last ...
"Tachycardia; St. Jude Medical Announces FDA Approval of Assura (TM) Implantable Defibrillators with Features That Reduce ...
They concluded that it produces suicide attempt, suicide; death; QTc prolongation, low blood pressure; tachycardia; sedation; ...
Tachycardia. This is caused by the decreased preload, decreased cardiac output, and leads to increased frequency. In pregnant ...
"Tachycardia, Fast Heart Rate". Tachycardia. American Heart Association. 2 May 2013. Retrieved 21 May 2014. Fuster, Wayne & ... Tachycardia is a high heart rate, defined as above 100 bpm at rest. Bradycardia is a low heart rate, defined as below 60 bpm at ... Tachycardia is defined as a resting heart rate above 100 bpm, though persistent rest rates between 80 and 100 bpm, mainly if ... Those techniques can assess the heart rate by measuring the delay between pulses.[citation needed] Tachycardia is a resting ...
Bramwell, C. (28 February 1953). "Tachycardia". Br Med J. 1 (480): 500-502. doi:10.1136/bmj.1.4808.500. PMC 2015392. PMID ...
... tachycardia; palpitation. Psychiatric: Confusional states (especially in the elderly) with hallucinations, disorientation, ...
... tachycardia; bizarre, and possibly violent behavior; dry skin; dry mouth; illusions; and severe mydriasis (dilated pupils) with ...
Ventricular tachycardia (VT) is a rapid heartbeat that starts in the lower chambers of the heart (ventricles). ... Ventricular tachycardia may not cause symptoms in some people. However, it can be deadly. It is a major cause of sudden cardiac ... Ventricular tachycardia (VT) is a rapid heartbeat that starts in the lower chambers of the heart (ventricles). ...
Ventricular tachycardia (VT) refers to any rhythm faster than 100 (or 120) beats/min arising distal to the bundle of His. The ... Any wide-complex tachycardia tracing should raise the possibility of ventricular tachycardia, but closer scrutiny confirms left ... Using the 12-lead ECG to localize the origin of atrial and ventricular tachycardias: part 2--ventricular tachycardia. J ... In patients with a prior MI, the most common mechanism of wide QRS complex tachycardia is ventricular tachycardia. ...
"Supraventricular Tachycardia, Junctional Ectopic Tachycardia: Overview - eMedicine". Retrieved 21 December 2008.. *^ a b ... Tachycardia (from the Greek takhys, meaning "swift", and kardia, meaning heart) means a swift heart rate.[11] ... Junctional ectopic tachycardia (JET) is a rare syndrome of the heart that manifests in patients recovering from heart surgery.[ ... This syndrome, which may also referred to as His bundle tachycardia, is resistant to therapy and can be difficult to treat.[3][ ...
Postural orthostatic tachycardia syndrome (POTS) happens when the autonomic nervous system - which controls things like heart ... What Is Postural Orthostatic Tachycardia Syndrome (POTS)?. Postural orthostatic tachycardia syndrome (POTS) is a disorder that ... The autonomic nervous system problems seen in POTS - also called postural tachycardia (ta-kih-KAR-dee-uh) syndrome - can affect ... What Are the Signs & Symptoms of Postural Orthostatic Tachycardia Syndrome (POTS)?. POTS is named for an unusual jump in the ...
Hi, I am 43 years old, 140lbs, 59, run 60-70 miles per week. About 3 weeks ago, I suffered from 60-hour diarrhea, which I finally controlled with immodium. I did not run for a few days and then tr...
... Aka: Delayed Tachycardia *Definition. *Normal Heart Rate despite impending cardiovascular collapse (e.g. ... Tachycardia is typically an early marker of volume depletion or shock. *In some cases, Heart Rate may be unreliable in ...
Ventricular tachycardia ablation, Implantable cardioverter-defibrillator insertion, Atrial tachycardia, Sudden cardiac arrest, ... Supraventricular tachycardia, Atrial flutter, Wolff-Parkinson-White syndrome, Heart palpitations, Ventricular tachycardia, ... diagnostic tests and treatments for supraventricular tachycardia and other heart conditions. Staff in the Heart Rhythm Clinic ...
... Heart Rhythm. 2014 Jun;11(6):1085-6. ...
... you may have sinus tachycardia. Your physician can help you determine the cause. ... Sinus tachycardia causes the heart to beat more than 100 beats per minute, which is considered too fast. You have probably ... Sinus tachycardia is diagnosed when the heart rate is quicker than normal, but the heart continues to beat properly. The ... Inappropriate sinus tachycardia may require treatment. Your physician will find and treat the cause for abnormal heart rhythm ...
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Atrial TachycardiaAtrial Tachycardia is an abnormal heartbeat that is caused by an abnormal firing of electrical signals in the ... Ventricular TachycardiaVentricular Tachycardia is an abnormal heartbeat that is caused by an abnormal firing of electrical ... Ventricular TachycardiaVentricular Tachycardia is an abnormal heartbeat that is caused by an abnormal firing of electrical ... Atrial Tachycardia is an abnormal heartbeat that is caused by an abnormal firing of electrical signals in the atria of the ...
The incidence of multifocal atrial tachycardia (MAT) is very low and accounts for less than 1% of su ... The incidence of multifocal atrial tachycardia (MAT) is very low and accounts for less than 1% of supraventricular tachycardia ... Multifocal Atrial Tachycardia in 2 Children. Hsieh, Ming-Yun; Lee, Pi-Chang*; Hwang, Betau; Meng, C Laura C. ... Multifocal Atrial Tachycardia in 2 Children : Journal of the Chinese Medical Association. ...
In my previous article, I described how to recognize preexcitation syndromes, including Wolff-Parkinson-White (WPW) syndrome, on an ECG. In this...
... photos and unpublished archives about Postural Orthostatic Tachycardia Syndrome, many of them with free online access. ...
Supraventricular Tachycardia. / Penny-Peterson, Erica D.; Naccarelli, Gerald V.. Evidence-Based Cardiology: Third Edition. ... Penny-Peterson, E. D., & Naccarelli, G. V. (2010). Supraventricular Tachycardia. In Evidence-Based Cardiology: Third Edition ( ... Supraventricular Tachycardia. In Evidence-Based Cardiology: Third Edition. Wiley-Blackwell. 2010. p. 606-618 doi: 10.1002/ ... Penny-Peterson, Erica D. ; Naccarelli, Gerald V. / Supraventricular Tachycardia. Evidence-Based Cardiology: Third Edition. ...
There are MANY conditions that cause tachycardia, one of them is called Postural Orthostatic Tachycardia Syndrome or POTS. I ... This article is false! The fact that someone has Tachycardia has nothing to do with standing and/or sitting!. Take it from ... Lisa, Postural Orthostatic Tachycardia Syndrome is primarily debilitating due to this part of it. Postural Orthostatic (meaning ... Just because you have tachycardia from something else doesnt mean that my condition doesnt exist. ...
tachycardia symptom/finding detailed information in Haz-Map database. ...
Learn about sinus tachycardia, including causes, risk factors, symptoms, diagnosis and treatment from the cardiology experts at ... Treatment for sinus tachycardia. In most cases, sinus tachycardia is not life-threatening and does not need to be treated. If ... Causes of sinus tachycardia. Sinus tachycardia is a type of irregular heartbeat that is characterized by a faster than normal ... What is Sinus Tachycardia?. Sinus tachycardia occurs when your hearts natural pacemaker, the sinoatrial node, sends quicker ...
In most cases, if you experience short episodes of supraventricular tachycardia (SVT) with no symptoms, your health care ...
... is the most common type of reentrant supraventricular tachycardia (SVT). The substrate for AVNRT is the presence of dual AV ... Atrioventricular Nodal Reentry Tachycardia. Atypical atrioventricular nodal (AV) reentry tachycardia. Often, an inverted P wave ... Atrioventricular Nodal Reentry Tachycardia. Typical atrioventricular nodal (AV) reentry tachycardia. In this electrocardiogram ... 2019 ESC/AEPC Guidelines for the Management of Supraventricular Tachycardia. Supraventricular Tachycardia Clinical Practice ...
Amiodarone management of junctional ectopic tachycardia after cardiac surgery in children. Message subject: (Your Name) has ...
Inappropriate Sinus Tachycardia. Management of inappropriate sinus tachycardia ▸ Beta blockers (Class I, level of evidence C). ... Focal and Nonparoxysmal Junctional Tachycardia. Management of focal and nonparoxysmal junctional tachycardia ... Focal junctional tachycardia ▸ Beta blockers (Class IIa, level of evidence C). OR. ▸ Amiodarone (Class IIa, level of evidence C ... Non paroxysmal junctional tachycardia ▸ Reverse digitalis toxicity (Class I, level of evidence C). AND. ▸ Correct hypokalemia ( ...
Our experts diagnose and treat tachycardia, a life-threatening arrhythmia that causes a fast heartbeat. We offer medications, ... Tachycardia (Fast Heart Rate) Tachycardia is a type of arrhythmia that causes your heart to beat too fast. It raises the risk ... What Is Tachycardia?. Tachycardia is a heart rate over 100 beats per minute when you are at rest. A fault in your hearts ... Tachycardia Treatments. The goal of tachycardia treatment is to control and regulate your heartbeat. To do so, we may also need ...
Objectives: Patients with postural tachycardia syndrome (POTS) often appear anxious and report inattention. Patients with POTS ...
ECG shows a regular atrial tachycardia with P-wave mo... ... Focal atrial tachycardia (focal AT) is characterised as a rapid ... Focal AT is a regular tachycardia and is often confused with other regular supraventricular tachycardias, specifically re-entry ... ECG shows a regular atrial tachycardia with P-wave morphology different from that in sinus tachycardia. ... For sustained tachycardias, cardiology consultation, class Ia/Ic or III anti-arrhythmic agents, and ablative therapy are ...
Nathan Almeida answered: No: Inappropriate sinus tachycardia does not cause atrial fibrillation, though the 2 may coexist. If ... Ecg shows sinus tachycardia(HR 129bpm). Echo are all normal except mild MR, mild TR/mild PAH with RVSP 45mmhg. Is mild PAH ... Sinus tachycardia heart rate 138. At home its always less than 100.doc ordered ecg because i had palpitations.i hv palp at home ... Inappropriate sinus tachycardia does not cause atrial fibrillation, though the 2 may coexist. If you did have a procedure such ...
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I havent been able to sleep because of my tachycardia. I have POTS so its effected by position. Last night I didnt fall ... I swear sometimes I feel like the albuterol (the last 7 years) gave me the tachycardia to begin with. But I know I have the ... I havent been able to sleep because of my tachycardia. I have POTS so its effected by position. Last night I didnt fall ... Sorry to hear about your problems...perhaps you need a beta blocker at night to help with the tachycardia. Otherwise, you may ...
Orthodeoxia and postural orthostatic tachycardia in patients with pulmonary arteriovenous malformations: a prospective 8-year ... Orthodeoxia and postural orthostatic tachycardia in patients with pulmonary arteriovenous malformations: a prospective 8-year ... Orthodeoxia and postural orthostatic tachycardia in patients with pulmonary arteriovenous malformations: a prospective 8-year ...
Weight gain Rashes Depression Fatigue Irritability Tachycardia Insomnia Vision Problems Hearing Loss Heart palpitations ... inproceedings{Barua2006WeightGR, title={Weight gain Rashes Depression Fatigue Irritability Tachycardia Insomnia Vision Problems ...
  • In this case the right bundle branch block was present during tachycardia and during normal sinus rhythm. (wikipedia.org)
  • Sinus tachycardia is diagnosed when the heart rate is quicker than normal, but the heart continues to beat properly. (uky.edu)
  • Sinus tachycardia causes the heart to beat more than 100 beats per minute, which is considered too fast. (uky.edu)
  • You have probably experienced sinus tachycardia when startled, anxious or after a hard workout. (uky.edu)
  • This is called inappropriate sinus tachycardia, and adults who experience this should seek evaluation from a healthcare professional. (uky.edu)
  • It is not always possible to prevent sinus tachycardia, but your healthcare provider can work with you to find and avoid triggers that lead to the abnormal heart rhythm. (uky.edu)
  • This test will help your healthcare provider determine if you have sinus tachycardia, or rule out a similar condition. (uky.edu)
  • Your provider will use this to detect inappropriate sinus tachycardia. (uky.edu)
  • Sinus tachycardia related to physical activity or emotional stress is usually resolved or improved by addressing the underlying condition. (uky.edu)
  • Inappropriate sinus tachycardia may require treatment. (uky.edu)
  • What is Sinus Tachycardia? (mercy.com)
  • Sinus tachycardia occurs when your heart's natural pacemaker, the sinoatrial node, sends quicker than standard signals, causing a rapid heart rate increase. (mercy.com)
  • The cause of sinus tachycardia is unknown but is thought to be caused by a problem with your sinus node, nerve dysfunction that causes your heart rate to decrease, or something triggers your heart rate to increase. (mercy.com)
  • Conditions that put a strain on the heart, such as diabetes, heart disease, anemia, or high blood pressure, increase your risk of developing inappropriate sinus tachycardia. (mercy.com)
  • The most common symptom of inappropriate sinus tachycardia is an inexplicably fast heart rate, even at rest. (mercy.com)
  • Sinus tachycardia is a type of irregular heartbeat that is characterized by a faster than normal heart rhythm. (mercy.com)
  • There are two types of sinus tachycardia - normal and inappropriate. (mercy.com)
  • While the exact cause of inappropriate sinus tachycardia is unknown, it can be caused by several issues. (mercy.com)
  • While inappropriate sinus tachycardia is not life-threatening for most people, you should schedule an appointment with your doctor to discuss your risk. (mercy.com)
  • Your doctor can diagnose inappropriate sinus tachycardia. (mercy.com)
  • In most cases, sinus tachycardia is not life-threatening and does not need to be treated. (mercy.com)
  • If an underlying condition is causing your sinus tachycardia, it needs to be treated. (mercy.com)
  • Making lifestyle changes such as eating a healthy diet, exercising, maintaining a healthy weight, and avoiding activities that increase your heart rate, can help reduce your risk of experiencing inappropriate sinus tachycardia. (mercy.com)
  • Narrow complex tachycardia may originate in the sinus node , atria , AV node , bundle of His , or a combination of these tissues. (wikidoc.org)
  • ECG shows a regular atrial tachycardia with P-wave morphology different from that in sinus tachycardia. (bmj.com)
  • Focal AT is a regular tachycardia and is often confused with other regular supraventricular tachycardias, specifically re-entry tachycardias, sinus tachycardia, and atrial flutter. (bmj.com)
  • Can sinus tachycardia lead to atrial fibrillation? (healthtap.com)
  • Can innapropriate sinus tachycardia lead to atrial fibrillation got to go to hospital monday for ecg my heart rhythm gone from normal to abnormal? (healthtap.com)
  • Inappropriate sinus tachycardia does not cause atrial fibrillation , though the 2 may coexist. (healthtap.com)
  • Sinus tachycardia heart rate 138. (healthtap.com)
  • In such cases, the development of the ventricular and sinus reciprocal tachycardia is not so frequent. (arrhythmia.center)
  • Note also that the change in the P-wave axis at the onset of tachycardia makes sinus tachycardia unlikely. (medscape.com)
  • An electrocardiogram reveals sinus tachycardia at 103, with diffuse upward ST segment elevation in most leads. (medpagetoday.com)
  • In patients with stable supraventricular tachycardia, modifying the Valsalva maneuver is significantly more effective in restoring sinus rhythm. (the-hospitalist.org)
  • I guess not having anymore bouts of afib helped, but last year I had to go to hospital twice for treatment for SVT, sinus ventricular tachycardia. (diabetesdaily.com)
  • SS declares that she has given lectures regionally and nationally that include paroxysmal atrial tachycardia. (bmj.com)
  • Supraventricular tachycardia (SVT), paroxysmal atrial tachycardia (PAT) or paroxysmal supraventricular tachycardia (PSVT). (chrichmond.org)
  • Our team has completed thousands of ablation procedures, including more than 1,500 ablation treatments for supraventricular tachycardia, a type of V-tach. (barnesjewish.org)
  • What are the determinants of ventricular tachycardia (VT) recurrence in patients who underwent VT ablation for post-infarction VT? (acc.org)
  • The National Heart Center in the Royal Hospital organized recently a training workshop on Ablation of Ventricular Tachycardia using Contact Force Catheter in the presence of Prof. Fasini, specialist in electrocardiogram from Italy along with participation of number of medical staff and allied medical professionals in the field of cardiac catheterization at the National Heart Center. (gov.om)
  • Pacing from the ablation catheter during VT terminates the tachycardia on the second stimulus without evidence of global capture, presumably by rendering the critical isthmus refractory at a time when it would be needed to conduct the next reentrant wave of depolarization. (elsevier.com)
  • The advent of three-dimensional (3-D) high-density and rapid electroanatomic mapping to characterize atrial tachycardias appears to have the potential to result in favorable outcomes following ablation. (medscape.com)
  • Ventricular tachycardia ablation in children. (bvsalud.org)
  • The ablation of ventricular tachycardia , including premature ventricular contractions , is an approved, albeit infrequent procedure in pediatric patients . (bvsalud.org)
  • The purpose of this study was to share a high-volume center experience and patient outcomes for catheter ablation of ventricular ectopy and ventricular tachycardia in pediatric population . (bvsalud.org)
  • Ventricular tachycardia/fibrillation ablation : the state of the art based on the VeniceChart international consensus document / edited by Andrea Natale, Antonio Raviele . (upol.cz)
  • This electrocardiogram (ECG) shows rapid monomorphic ventricular tachycardia (VT), 280 beats/min, associated with hemodynamic collapse. (medscape.com)
  • During the workshop, Dr. Najeeb al-Rawahi, Senior Consultant in electrocardiogram and Dr. Ismail al-Abri, Consultant in electrocardiogram at the National Heart Centre performed number of catheters for patients suffering from Ventricular Tachycardia by the three-dimensions imaging. (gov.om)
  • An electrocardiogram is a very common type of test that is mainly used for detecting tachycardia. (epainassist.com)
  • Rhythmic tachycardia is expressed in a rapid heartbeat, with which the RR intervals are displayed with a small variation on the electrocardiogram. (arrhythmia.center)
  • A 12-lead electrocardiogram (ECG) and rhythm strip is an important tool to help identify, locate, and differentiate atrial tachycardia. (medscape.com)
  • Amiodarone management of junctional ectopic tachycardia after cardiac surgery in children. (bmj.com)
  • This invention relates to implanted cardiac stimulators, and more particularly, to implanated cardiac stimulators which detect the occurrence of tachycardia and derive one or more stimulating pulses in a temporal relationship to naturally occurring cardiac stimulus to arrest the tachycardia. (justia.com)
  • The workshop aimed to train the medical staff on the best ways to conduct catheter for ventricular tachycardia by the three-dimensions imaging used for the first time in the Sultanate, acquaint them with the latest treatment and diagnostic methods in the field of cardiac catheterization, as well as exchange experiences and views on the basics of cardiac catheterization. (gov.om)
  • Tachycardia is a very common type of cardiac rhythm disorder in which it is seen that the resting heart rate becomes faster than what it should have normally. (epainassist.com)
  • Cardiac MRI is another useful test that is mostly performed for detecting tachycardia. (epainassist.com)
  • All patients who present acutely with possible atrial tachycardia should be placed on pulse oximetry and a cardiac monitor. (medscape.com)
  • Chest radiography is indicated to evaluate for a pulmonary etiology (eg, chronic obstructive pulmonary disease) and to delineate cardiac size and structures and cardiac findings in patients who present with tachycardia-induced cardiomyopathy and in those with complex congenital heart disease. (medscape.com)
  • Cardiac monitoring in the bay revealed ventricular tachycardia (a heart rhythm incompatible with life), and cardiopulmonary resuscitation (CPR) and advanced life support (ALS) were begun. (cdc.gov)
  • [ 17 ] released guidelines for the management of supraventricular tachycardia which include specific recommendations for both acute and ongoing management of atrioventricular node reentry tachycardia (AVNRT). (medscape.com)
  • This condition is typically called reentry tachycardia. (justia.com)
  • If the conduction velocity of the pathway and the refractory period of the heart tissue obtain a critical relationship, a reentry tachycardia may be initiated. (justia.com)
  • Accordingly, if the signal completes its circus movement as the muscle concludes the refractory period, a reentry path is established and a condition of tachycardia now exists. (justia.com)
  • It has been found that a tachycardia based upon reentry may be stopped by refractory tissue in the reentry pathway. (justia.com)
  • The different findings during electrophysiology studies may help guide the electrophysiologist in determining whether the mechanism of ventricular tachycardia (VT) is focal or macro-reentry. (elsevier.com)
  • Automatic or triggered atrial tachycardia generally looks distinctly different, but it may have similar features to typical atrioventricular nodal reentry tachycardia (AVNRT) if it originates in the low atrial septal area and if 1:1 conduction occurs. (medscape.com)
  • Brubaker S, Long B, Koyfman A. Alternative treatment options for atrioventricular-nodal-reentry tachycardia: an emergency medicine review. (medscape.com)
  • These features essentially exclude atrioventricular nodal reentry tachycardia and atrioventricular tachycardia via an accessory pathway. (medscape.com)
  • If you have signs or symptoms of tachycardia, you will need additional testing to determine the type of tachycardia you have. (mercy.com)
  • Apart from that, doctor, can find out the type of tachycardia that the patient is suffering from. (epainassist.com)
  • You may not experience any symptoms of tachycardia. (barnesjewish.org)
  • Knowing about the symptoms of tachycardia and the ways in which it is diagnosed can help a great deal in this matter. (epainassist.com)
  • Postural modification to the standard Valsalva manoeuvre for emergency treatment of supraventricular tachycardias (REVERT): a randomised controlled trial [published online ahead of print August 24, 2015]. (the-hospitalist.org)
  • Patients of heart surgery may experience an accelerated narrow complex tachycardia, usually within the first 24-48 hours (but occasionally longer) after surgery. (wikipedia.org)
  • The diagnosis of narrow complex tachycardia is based on the ECG findings. (wikidoc.org)
  • Junctional ectopic tachycardia (JET) is a rare syndrome of the heart that manifests in patients recovering from heart surgery. (wikipedia.org)
  • Mayo Clinic doctors and surgeons trained in heart rhythm disorders study genetics, causes, diagnostic tests and treatments for supraventricular tachycardia and other heart conditions. (mayoclinic.org)
  • The differential diagnosis of JET includes other forms of supraventricular tachycardia , most commonly atrioventricular nodal reentrant tachycardia (AVNRT). (wikipedia.org)
  • In orthodromic tachycardias (ie, due to a bypass tract), the P wave typically falls distinctly after the QRS complex, in contrast to the superimposition most commonly seen with AVNRT. (medscape.com)
  • Orthodromic AV-reciprocating tachycardia (AVRT) tends to occur at an earlier age and more frequently in males than AVNRT. (medscape.com)
  • In August 2019, the European Society of Cardiology (ESC) in collaboration with the Association for European Paediatric and Congenital Cardiology (AEPC) released recommendations on the management of supraventricular tachycardia. (medscape.com)
  • For sustained tachycardias, cardiology consultation, class Ia/Ic or III anti-arrhythmic agents, and ablative therapy are appropriate. (bmj.com)
  • 2019 ESC Guidelines for the management of patients with supraventricular tachycardiaThe Task Force for the management of patients with supraventricular tachycardia of the European Society of Cardiology (ESC). (bmj.com)
  • Postural orthostatic tachycardia syndrome (POTS) is a disorder that can make someone feel faint or dizzy. (kidshealth.org)
  • The autonomic nervous system problems seen in POTS - also called postural tachycardia (ta-kih-KAR-dee-uh) syndrome - can affect children and adults. (kidshealth.org)
  • What Are the Signs & Symptoms of Postural Orthostatic Tachycardia Syndrome (POTS)? (kidshealth.org)
  • Patients with postural tachycardia syndrome (POTS) often appear anxious and report inattention. (bmj.com)
  • Aug. 6, 2015-Vanderbilt University Medical Center (VUMC) and Dysautonomia International are partnering to launch the first large international study on postural orthostatic tachycardia syndrome (POTS), which impacts an estimated 500,000 to 3 million patients in the United States and millions more around the globe. (vumc.org)
  • My son was diagnosed with Postural Orthostatic Tachycardia Syndrome (POTS) in October 2011. (latitudes.org)
  • Multifocal atrial tachycardia is often associated with pulmonary disease or medications such as digoxin. (medscape.com)
  • On the 11th of February 2021 at 5 PM CET (11 AM EST), Finapres Medical Systems sponsored the webinar "Postural Orthostatic Tachycardia Syndrome - Overview and Focus on Non-Pharmacological Approaches" , hosted by Inside Scientific! (finapres.com)
  • Postural orthostatic tachycardia syndrome is reported only by a few people who take Acetylcysteine. (ehealthme.com)
  • The phase IV clinical study analyzes which people take Acetylcysteine and have Postural orthostatic tachycardia syndrome. (ehealthme.com)
  • Among them, 2 people (0.02%) have Postural orthostatic tachycardia syndrome. (ehealthme.com)
  • Postural orthostatic tachycardia syndrome (abnormal increase in heart rate on becoming upright) is found to be associated with 500 drugs and 244 conditions by eHealthMe. (ehealthme.com)
  • Do you take Acetylcysteine and have Postural orthostatic tachycardia syndrome? (ehealthme.com)
  • Orthostatic Intolerance and Postural Orthostatic Tachycardia Syndrome in Joint Hypermobility Syndrome/Ehlers-Danlos Syndrome, Hypermobility Type: Neurovegetative Dysregulation or Autonomic Failure? (ehlers-danlos.com)
  • At tilt, 48.6% patients showed postural orthostatic tachycardia, 31.4% orthostatic intolerance, 20% normal results. (ehlers-danlos.com)
  • My son was unable to stand or walk, suffered with severe scalp pain, headache, hoarse voice, brain fog, hypersensitive to touch, and was also diagnosed with Postural Orthostatic Tachycardia Syndrome. (latitudes.org)
  • This syndrome, which may also referred to as His bundle tachycardia, is resistant to therapy and can be difficult to treat. (wikipedia.org)
  • Tachycardia is a type of arrhythmia that causes your heart to beat too fast. (barnesjewish.org)
  • Yet another arrhythmia with serious consequences is tachycardia, a condition where an abnormally high heart beat rate occurs. (justia.com)
  • Their heartbeat can become very fast and irregular, causing an arrhythmia called ventricular tachycardia (VT). (nsw.gov.au)
  • One thing is for sure - the treatment options will depend on the cause of tachycardia . (thehealthblog.net)
  • A second cause of tachycardia involves a rapidly circulating impulse in a circus pathway. (justia.com)
  • Ventricular tachycardia (VT) is a rapid heartbeat that starts in the lower chambers of the heart (ventricles). (medlineplus.gov)
  • The appearance is usually of a tachycardia with rapid, regular ventricular rates of 170-260 beats per minute. (wikipedia.org)
  • Focal atrial tachycardia (focal AT) is characterised as a rapid regular rhythm arising from a discrete area within the atria. (bmj.com)
  • Mar. 13, 2014-Circulating "autoantibodies" possibly triggered by a viral illness may contribute an abnormally rapid heart rate or tachycardia upon standing that affects 500,000 Americans, mostly young women, according to researchers at the University of Oklahoma and Vanderbilt University. (vumc.org)
  • When there is a rapid heartbeat, you do not always have to think about a pathological tachycardia immediately. (arrhythmia.center)
  • In young males, AVRT tends to be a more rapid supraventricular tachycardia (SVT) than are most AVNRTs. (medscape.com)
  • Atrial Tachycardia is an abnormal heartbeat that is caused by an abnormal firing of electrical signals in the atria of the heart that interfere with electrical signals coming from the natural pacemaker of the heart (SA node). (medmovie.com)
  • The goal of tachycardia treatment is to control and regulate your heartbeat. (barnesjewish.org)
  • Metoprolol treats an abnormally fast heartbeat, known as supraventricular tachycardia (SVT), by slowing heart rate. (druggenius.com)
  • Computerized Tomography (CT) Scan for Diagnosing Tachycardia. (epainassist.com)
  • One of the main complications of pathological tachycardia is the possibility of developing cardiomyopathy which can lead to heart failure. (epainassist.com)
  • When determining with rhythmic tachycardia electrocardiography, it is necessary to carry out appropriate treatment, as well as in the case of other forms of rhythm disturbance. (arrhythmia.center)
  • The incidence of multifocal atrial tachycardia (MAT) is very low and accounts for less than 1% of supraventricular tachycardia in infants and children. (lww.com)
  • What's causes atrial tachycardia , i'm 30 f started noticing fast heart in july hosp first thought was cardiomyopathy according to my ecgs? (healthtap.com)
  • Tachycardia-induced cardiomyopathy (TIC) is a reversible cause of LV systolic dysfunction that can complicate any supraventricular or ventricular tachyarrhythmia. (uct.ac.za)
  • methods Treatment of rhythmic tachycardia heart in men, women and children. (arrhythmia.center)
  • Before starting treatment for rhythmic tachycardia, it is important to determine the underlying cause of its development. (arrhythmia.center)
  • Tachycardias : mechanisms, diagnosis, treatment / [edited by] Mark E. Josephson, Hein J. J. Wellens. (who.int)
  • Treatment is typically needed if the tachycardia occurs often, lasts long or interferes with your child's normal activities. (chrichmond.org)
  • Thus, one of the easiest ways in which you would know if you have tachycardia and get a diagnosis from the physician. (epainassist.com)
  • ECG features to consider in the diagnosis of atrial tachycardia include P wave morphology and axis (see the image below), PR interval, and PP interval variations. (medscape.com)
  • The efficacy of performing vaginal tests or the administration of adenosine may indicate the localization of an ectopic focal point that facilitates the appearance of tachycardia. (arrhythmia.center)
  • This is a focal tachycardia originating in the atria. (medscape.com)
  • Sustained ventricular tachycardia in a paediatric patient with acute COVID-19 myocarditis. (duke.edu)
  • Herein, we present the first paediatric case of refractory ventricular tachycardia from acute fulminant myocarditis secondary to acute COVID-19 infection. (duke.edu)
  • Changes in frequency domain measures of heart rate variability in relation to the onset of ventricular tachycardia in acute myocardial infarction. (arctichealth.org)
  • We analysed the power spectral components of total power, very low frequency power (0.0033-0.04 Hz), low frequency (0.04-0.15 Hz) and high frequency (0.15-0.40 Hz) power in 12 consecutive patients accompanied with 27 episodes of ventricular tachycardia in acute myocardial infarction. (arctichealth.org)
  • Four hundred thirty-three patients with stable supraventricular tachycardia (excluding atrial fibrillation or flutter) were randomized to use the Valsalva maneuver (control) or modified Valsalva maneuver (intervention). (the-hospitalist.org)
  • Efficacy and safety of moricizine in patients with ventricular tachycardia: results of a placebo-controlled prospective long-term clinical trial. (wikigenes.org)
  • Rapport d'activités 2022 de l'OMS Niger : principaux résultats de l'OMS atteints en étroite collaboration avec le Gouvernement, les partenaires et d'autres acteurs. (who.int)
  • 1 Hence, early recognition is important to manage tachycardia well and prevent complications. (epainassist.com)
  • In the absence of an organic pathology of the heart, rhythmic tachycardia does not cause serious complications. (arrhythmia.center)
  • Symptoms of inappropriate tachycardia can mimic symptoms from other heart conditions, so it is essential to schedule an appointment with your doctor to diagnose the underlying cause of your symptoms. (mercy.com)
  • Tachycardia is a heart rate over 100 beats per minute when you are at rest. (barnesjewish.org)
  • We can often stop ventricular tachycardia with a short electrical shock to the heart. (barnesjewish.org)
  • Have done echo heart, ecg, stresstest, chest xray, doc found ectopic atrial tachycardia.It doesn't xplain my pain and high pulse when eat not present? (healthtap.com)
  • Tachycardia severly affects the ability of the heart to pump blood and the higher the heart beat rate, the more dangerous the condition. (justia.com)
  • Catecholaminergic Polymorphic Ventricular Tachycardia (CPVT) is a rare heart rhythm disorder, which can cause periods of abnormally fast heart rhythm called arrhythmias. (nsw.gov.au)
  • Increased heart rate happens when there is any abnormality in the heart and that condition is called tachycardia. (epainassist.com)
  • With the help of these X-ray images, the doctors can study the heart through the detailed cross-sectional view of it and thereby they can come to the conclusion whether the patient has tachycardia or not. (epainassist.com)
  • A patient is having high fever, tachycardia and sweating. (testpreppractice.net)
  • Fever, tachycardia, and tachypnea have been seen upon clinical examination. (cdc.gov)
  • The patient took amiodarone regularly, and no tachycardia was detected during the follow-up period. (lww.com)
  • A second pulse is then applied in a time interval following the refractory interval pulse to break the tachycardia. (justia.com)
  • As hereinbelow discussed, it is known that the arrival of a single stimulating pulse at a critical moment may terminate the tachycardia. (justia.com)
  • Control of tachycardia has generally occurred in a clinical situation where the stimulating pulse generator is located externally of the patient so that stimulating pulse timing can be varied. (justia.com)
  • Where a single pulse has been employed, the pulse frequency has been chosen either greater than or lesser than the naturally occurring tachycardia whereby the applied pulses occur randomly between tachycardia beats until a pulse happens to occur at the critical point to block the tachycardia. (justia.com)
  • In some instances, an extremely high pulse rate may be effective to terminate the tachycardia. (justia.com)
  • In either event, the timing of the pulse is random with respect to the tachycardia. (justia.com)
  • The response to adenosine in JET is a temporary loss of conduction to the atria leading to continuation of the tachycardia but with atrioventricular dissociation. (wikipedia.org)