Abnormally rapid heartbeat, usually with a HEART RATE above 100 beats per minute for adults. Tachycardia accompanied by disturbance in the cardiac depolarization (cardiac arrhythmia) is called tachyarrhythmia.
An abnormally rapid ventricular rhythm usually in excess of 150 beats per minute. It is generated within the ventricle below the BUNDLE OF HIS, either as autonomic impulse formation or reentrant impulse conduction. Depending on the etiology, onset of ventricular tachycardia can be paroxysmal (sudden) or nonparoxysmal, its wide QRS complexes can be uniform or polymorphic, and the ventricular beating may be independent of the atrial beating (AV dissociation).
A generic expression for any tachycardia that originates above the BUNDLE OF HIS.
Abnormally rapid heartbeats caused by reentry of atrial impulse into the dual (fast and slow) pathways of ATRIOVENTRICULAR NODE. The common type involves a blocked atrial impulse in the slow pathway which reenters the fast pathway in a retrograde direction and simultaneously conducts to the atria and the ventricles leading to rapid HEART RATE of 150-250 beats per minute.
Abnormally rapid heartbeats with sudden onset and cessation.
Abnormally rapid heartbeats originating from one or more automatic foci (nonsinus pacemakers) in the HEART ATRIUM but away from the SINOATRIAL NODE. Unlike the reentry mechanism, automatic tachycardia speeds up and slows down gradually. The episode is characterized by a HEART RATE between 135 to less than 200 beats per minute and lasting 30 seconds or longer.
Simple rapid heartbeats caused by rapid discharge of impulses from the SINOATRIAL NODE, usually between 100 and 180 beats/min in adults. It is characterized by a gradual onset and termination. Sinus tachycardia is common in infants, young children, and adults during strenuous physical activities.
Removal of tissue with electrical current delivered via electrodes positioned at the distal end of a catheter. Energy sources are commonly direct current (DC-shock) or alternating current at radiofrequencies (usually 750 kHz). The technique is used most often to ablate the AV junction and/or accessory pathways in order to interrupt AV conduction and produce AV block in the treatment of various tachyarrhythmias.
Recording of the moment-to-moment electromotive forces of the HEART as projected onto various sites on the body's surface, delineated as a scalar function of time. The recording is monitored by a tracing on slow moving chart paper or by observing it on a cardioscope, which is a CATHODE RAY TUBE DISPLAY.
A rare form of supraventricular tachycardia caused by automatic, not reentrant, conduction initiated from sites at the atrioventricular junction, but not the ATRIOVENTRICULAR NODE. It usually occurs during myocardial infarction, after heart surgery, or in digitalis intoxication with a HEART RATE ranging from 140 to 250 beats per minute.
A syndrome of ORTHOSTATIC INTOLERANCE combined with excessive upright TACHYCARDIA, and usually without associated ORTHOSTATIC HYPOTENSION. All variants have in common an excessively reduced venous return to the heart (central HYPOVOLEMIA) while upright.
Abnormally rapid heartbeats caused by reentrant conduction over the accessory pathways between the HEART ATRIA and the HEART VENTRICLES. The impulse can also travel in the reverse direction, as in some cases, atrial impulses travel to the ventricles over the accessory pathways and back to the atria over the BUNDLE OF HIS and the ATRIOVENTRICULAR NODE.
Abnormally rapid heartbeats caused by reentry circuit in or around the SINOATRIAL NODE. It is characterized by sudden onset and offset episodes of tachycardia with a HEART RATE of 100-150 beats per minute. The P wave is identical to the sinus P wave but with a longer PR interval.
An impulse-conducting system composed of modified cardiac muscle, having the power of spontaneous rhythmicity and conduction more highly developed than the rest of the heart.
Methods to induce and measure electrical activities at specific sites in the heart to diagnose and treat problems with the heart's electrical system.
Regulation of the rate of contraction of the heart muscles by an artificial pacemaker.
Agents used for the treatment or prevention of cardiac arrhythmias. They may affect the polarization-repolarization phase of the action potential, its excitability or refractoriness, or impulse conduction or membrane responsiveness within cardiac fibers. Anti-arrhythmia agents are often classed into four main groups according to their mechanism of action: sodium channel blockade, beta-adrenergic blockade, repolarization prolongation, or calcium channel blockade.
A small nodular mass of specialized muscle fibers located in the interatrial septum near the opening of the coronary sinus. It gives rise to the atrioventricular bundle of the conduction system of the heart.
Small band of specialized CARDIAC MUSCLE fibers that originates in the ATRIOVENTRICULAR NODE and extends into the membranous part of the interventricular septum. The bundle of His, consisting of the left and the right bundle branches, conducts the electrical impulses to the HEART VENTRICLES in generation of MYOCARDIAL CONTRACTION.
Recording of regional electrophysiological information by analysis of surface potentials to give a complete picture of the effects of the currents from the heart on the body surface. It has been applied to the diagnosis of old inferior myocardial infarction, localization of the bypass pathway in Wolff-Parkinson-White syndrome, recognition of ventricular hypertrophy, estimation of the size of a myocardial infarct, and the effects of different interventions designed to reduce infarct size. The limiting factor at present is the complexity of the recording and analysis, which requires 100 or more electrodes, sophisticated instrumentation, and dedicated personnel. (Braunwald, Heart Disease, 4th ed)
Any disturbances of the normal rhythmic beating of the heart or MYOCARDIAL CONTRACTION. Cardiac arrhythmias can be classified by the abnormalities in HEART RATE, disorders of electrical impulse generation, or impulse conduction.
A form of ventricular pre-excitation characterized by a short PR interval and a long QRS interval with a delta wave. In this syndrome, atrial impulses are abnormally conducted to the HEART VENTRICLES via an ACCESSORY CONDUCTING PATHWAY that is located between the wall of the right or left atria and the ventricles, also known as a BUNDLE OF KENT. The inherited form can be caused by mutation of PRKAG2 gene encoding a gamma-2 regulatory subunit of AMP-activated protein kinase.
A potentially lethal cardiac arrhythmia that is characterized by uncoordinated extremely rapid firing of electrical impulses (400-600/min) in HEART VENTRICLES. Such asynchronous ventricular quivering or fibrillation prevents any effective cardiac output and results in unconsciousness (SYNCOPE). It is one of the major electrocardiographic patterns seen with CARDIAC ARREST.
The number of times the HEART VENTRICLES contract per unit of time, usually per minute.
Implantable devices which continuously monitor the electrical activity of the heart and automatically detect and terminate ventricular tachycardia (TACHYCARDIA, VENTRICULAR) and VENTRICULAR FIBRILLATION. They consist of an impulse generator, batteries, and electrodes.
An antianginal and class III antiarrhythmic drug. It increases the duration of ventricular and atrial muscle action by inhibiting POTASSIUM CHANNELS and VOLTAGE-GATED SODIUM CHANNELS. There is a resulting decrease in heart rate and in vascular resistance.
Method in which prolonged electrocardiographic recordings are made on a portable tape recorder (Holter-type system) or solid-state device ("real-time" system), while the patient undergoes normal daily activities. It is useful in the diagnosis and management of intermittent cardiac arrhythmias and transient myocardial ischemia.
A potent anti-arrhythmia agent, effective in a wide range of ventricular and atrial ARRHYTHMIAS and TACHYCARDIAS.
A type of cardiac arrhythmia with premature contractions of the HEART VENTRICLES. It is characterized by the premature QRS complex on ECG that is of abnormal shape and great duration (generally >129 msec). It is the most common form of all cardiac arrhythmias. Premature ventricular complexes have no clinical significance except in concurrence with heart diseases.
The use of freezing as a special surgical technique to destroy or excise tissue.
The chambers of the heart, to which the BLOOD returns from the circulation.
A class Ia antiarrhythmic drug that is structurally-related to PROCAINE.
The innermost layer of the heart, comprised of endothelial cells.
Rapid, irregular atrial contractions caused by a block of electrical impulse conduction in the right atrium and a reentrant wave front traveling up the inter-atrial septum and down the right atrial free wall or vice versa. Unlike ATRIAL FIBRILLATION which is caused by abnormal impulse generation, typical atrial flutter is caused by abnormal impulse conduction. As in atrial fibrillation, patients with atrial flutter cannot effectively pump blood into the lower chambers of the heart (HEART VENTRICLES).
Impaired conduction of cardiac impulse that can occur anywhere along the conduction pathway, such as between the SINOATRIAL NODE and the right atrium (SA block) or between atria and ventricles (AV block). Heart blocks can be classified by the duration, frequency, or completeness of conduction block. Reversibility depends on the degree of structural or functional defects.
A form of heart block in which the electrical stimulation of HEART VENTRICLES is interrupted at either one of the branches of BUNDLE OF HIS thus preventing the simultaneous depolarization of the two ventricles.
A group of conditions in which HEART VENTRICLE activation by the atrial impulse is faster than the normal impulse conduction from the SINOATRIAL NODE. In these pre-excitation syndromes, atrial impulses often bypass the ATRIOVENTRICULAR NODE delay and travel via ACCESSORY CONDUCTING PATHWAYS connecting the atrium directly to the BUNDLE OF HIS.
Extra impulse-conducting tissue in the heart that creates abnormal impulse-conducting connections between HEART ATRIA and HEART VENTRICLES.
The lower right and left chambers of the heart. The right ventricle pumps venous BLOOD into the LUNGS and the left ventricle pumps oxygenated blood into the systemic arterial circulation.
A group of cardiac arrhythmias in which the cardiac contractions are not initiated at the SINOATRIAL NODE. They include both atrial and ventricular premature beats, and are also known as extra or ectopic heartbeats. Their frequency is increased in heart diseases.
The study of the generation and behavior of electrical charges in living organisms particularly the nervous system and the effects of electricity on living organisms.
Unexpected rapid natural death due to cardiovascular collapse within one hour of initial symptoms. It is usually caused by the worsening of existing heart diseases. The sudden onset of symptoms, such as CHEST PAIN and CARDIAC ARRHYTHMIAS, particularly VENTRICULAR TACHYCARDIA, can lead to the loss of consciousness and cardiac arrest followed by biological death. (from Braunwald's Heart Disease: A Textbook of Cardiovascular Medicine, 7th ed., 2005)
An electrical current applied to the HEART to terminate a disturbance of its rhythm, ARRHYTHMIAS, CARDIAC. (Stedman, 25th ed)
A transient loss of consciousness and postural tone caused by diminished blood flow to the brain (i.e., BRAIN ISCHEMIA). Presyncope refers to the sensation of lightheadedness and loss of strength that precedes a syncopal event or accompanies an incomplete syncope. (From Adams et al., Principles of Neurology, 6th ed, pp367-9)
Recording the locations and measurements of electrical activity in the EPICARDIUM by placing electrodes on the surface of the heart to analyze the patterns of activation and to locate arrhythmogenic sites.
Studies in which individuals or populations are followed to assess the outcome of exposures, procedures, or effects of a characteristic, e.g., occurrence of disease.
Evaluation undertaken to assess the results or consequences of management and procedures used in combating disease in order to determine the efficacy, effectiveness, safety, and practicability of these interventions in individual cases or series.
An adrenergic beta-antagonist that is used in the treatment of life-threatening arrhythmias.
Abnormal cardiac rhythm that is characterized by rapid, uncoordinated firing of electrical impulses in the upper chambers of the heart (HEART ATRIA). In such case, blood cannot be effectively pumped into the lower chambers of the heart (HEART VENTRICLES). It is caused by abnormal impulse generation.
Abnormal accumulation of serous fluid in two or more fetal compartments, such as SKIN; PLEURA; PERICARDIUM; PLACENTA; PERITONEUM; AMNIOTIC FLUID. General fetal EDEMA may be of non-immunologic origin, or of immunologic origin as in the case of ERYTHROBLASTOSIS FETALIS.
A congenital cardiomyopathy that is characterized by infiltration of adipose and fibrous tissue into the RIGHT VENTRICLE wall and loss of myocardial cells. Primary injuries usually are at the free wall of right ventricular and right atria resulting in ventricular and supraventricular arrhythmias.
The return of a sign, symptom, or disease after a remission.
A group of diseases in which the dominant feature is the involvement of the CARDIAC MUSCLE itself. Cardiomyopathies are classified according to their predominant pathophysiological features (DILATED CARDIOMYOPATHY; HYPERTROPHIC CARDIOMYOPATHY; RESTRICTIVE CARDIOMYOPATHY) or their etiological/pathological factors (CARDIOMYOPATHY, ALCOHOLIC; ENDOCARDIAL FIBROELASTOSIS).
The domestic dog, Canis familiaris, comprising about 400 breeds, of the carnivore family CANIDAE. They are worldwide in distribution and live in association with people. (Walker's Mammals of the World, 5th ed, p1065)
A device designed to stimulate, by electric impulses, contraction of the heart muscles. It may be temporary (external) or permanent (internal or internal-external).
A conical fibro-serous sac surrounding the HEART and the roots of the great vessels (AORTA; VENAE CAVAE; PULMONARY ARTERY). Pericardium consists of two sacs: the outer fibrous pericardium and the inner serous pericardium. The latter consists of an outer parietal layer facing the fibrous pericardium, and an inner visceral layer (epicardium) resting next to the heart, and a pericardial cavity between these two layers.
Drugs that bind to but do not activate beta-adrenergic receptors thereby blocking the actions of beta-adrenergic agonists. Adrenergic beta-antagonists are used for treatment of hypertension, cardiac arrhythmias, angina pectoris, glaucoma, migraine headaches, and anxiety.
The study of the electrical activity and characteristics of the HEART; MYOCARDIUM; and CARDIOMYOCYTES.
Elements of limited time intervals, contributing to particular results or situations.
An antiarrhythmia agent that is particularly effective in ventricular arrhythmias. It also has weak beta-blocking activity.
Cardiac arrhythmias that are characterized by excessively slow HEART RATE, usually below 50 beats per minute in human adults. They can be classified broadly into SINOATRIAL NODE dysfunction and ATRIOVENTRICULAR BLOCK.
PRESSURE of the BLOOD on the ARTERIES and other BLOOD VESSELS.
A form of ventricular pre-excitation characterized by a normal PR interval and a long QRS interval with an initial slow deflection (delta wave). In this syndrome, the atrial impulse travel to the ventricle via the MAHAIM FIBERS which connect ATRIOVENTRICULAR NODE directly to the right ventricle wall (NODOVENTRICULAR ACCESSORY PATHWAY) or to the RIGHT BUNDLE BRANCH OF HIS (nodofascicular accessory pathway).
A calcium channel blocker that is a class IV anti-arrhythmia agent.
The hollow, muscular organ that maintains the circulation of the blood.
Impaired impulse conduction from HEART ATRIA to HEART VENTRICLES. AV block can mean delayed or completely blocked impulse conduction.
A widely used non-cardioselective beta-adrenergic antagonist. Propranolol has been used for MYOCARDIAL INFARCTION; ARRHYTHMIA; ANGINA PECTORIS; HYPERTENSION; HYPERTHYROIDISM; MIGRAINE; PHEOCHROMOCYTOMA; and ANXIETY but adverse effects instigate replacement by newer drugs.
The movement and the forces involved in the movement of the blood through the CARDIOVASCULAR SYSTEM.
NECROSIS of the MYOCARDIUM caused by an obstruction of the blood supply to the heart (CORONARY CIRCULATION).
A cardiotonic glycoside obtained mainly from Digitalis lanata; it consists of three sugars and the aglycone DIGOXIGENIN. Digoxin has positive inotropic and negative chronotropic activity. It is used to control ventricular rate in ATRIAL FIBRILLATION and in the management of congestive heart failure with atrial fibrillation. Its use in congestive heart failure and sinus rhythm is less certain. The margin between toxic and therapeutic doses is small. (From Martindale, The Extra Pharmacopoeia, 30th ed, p666)
The small mass of modified cardiac muscle fibers located at the junction of the superior vena cava (VENA CAVA, SUPERIOR) and right atrium. Contraction impulses probably start in this node, spread over the atrium (HEART ATRIUM) and are then transmitted by the atrioventricular bundle (BUNDLE OF HIS) to the ventricle (HEART VENTRICLE).
Antiarrhythmic agent pharmacologically similar to LIDOCAINE. It may have some anticonvulsant properties.
A standard and widely accepted diagnostic test used to identify patients who have a vasodepressive and/or cardioinhibitory response as a cause of syncope. (From Braunwald, Heart Disease, 7th ed)
Studies used to test etiologic hypotheses in which inferences about an exposure to putative causal factors are derived from data relating to characteristics of persons under study or to events or experiences in their past. The essential feature is that some of the persons under study have the disease or outcome of interest and their characteristics are compared with those of unaffected persons.
A condition in which HEART VENTRICLES exhibit impaired function.
Procedures using an electrically heated wire or scalpel to treat hemorrhage (e.g., bleeding ulcers) and to ablate tumors, mucosal lesions, and refractory arrhythmias. It is different from ELECTROSURGERY which is used more for cutting tissue than destroying and in which the patient is part of the electric circuit.
Acidic protein found in SARCOPLASMIC RETICULUM that binds calcium to the extent of 700-900 nmoles/mg. It plays the role of sequestering calcium transported to the interior of the intracellular vesicle.
Ultrasonic recording of the size, motion, and composition of the heart and surrounding tissues. The standard approach is transthoracic.
Procedures in which placement of CARDIAC CATHETERS is performed for therapeutic or diagnostic procedures.
The abrupt cessation of all vital bodily functions, manifested by the permanent loss of total cerebral, respiratory, and cardiovascular functions.
Isopropyl analog of EPINEPHRINE; beta-sympathomimetic that acts on the heart, bronchi, skeletal muscle, alimentary tract, etc. It is used mainly as bronchodilator and heart stimulant.
The hemodynamic and electrophysiological action of the RIGHT ATRIUM.
Pathophysiological conditions of the FETUS in the UTERUS. Some fetal diseases may be treated with FETAL THERAPIES.
A form of CARDIAC MUSCLE disease that is characterized by ventricular dilation, VENTRICULAR DYSFUNCTION, and HEART FAILURE. Risk factors include SMOKING; ALCOHOL DRINKING; HYPERTENSION; INFECTION; PREGNANCY; and mutations in the LMNA gene encoding LAMIN TYPE A, a NUCLEAR LAMINA protein.
The period of time following the triggering of an ACTION POTENTIAL when the CELL MEMBRANE has changed to an unexcitable state and is gradually restored to the resting (excitable) state. During the absolute refractory period no other stimulus can trigger a response. This is followed by the relative refractory period during which the cell gradually becomes more excitable and the stronger impulse that is required to illicit a response gradually lessens to that required during the resting state.
Observation of a population for a sufficient number of persons over a sufficient number of years to generate incidence or mortality rates subsequent to the selection of the study group.
The fibrous tissue that replaces normal tissue during the process of WOUND HEALING.
Cessation of heart beat or MYOCARDIAL CONTRACTION. If it is treated within a few minutes, heart arrest can be reversed in most cases to normal cardiac rhythm and effective circulation.
A condition that is characterized by episodes of fainting (SYNCOPE) and varying degree of ventricular arrhythmia as indicated by the prolonged QT interval. The inherited forms are caused by mutation of genes encoding cardiac ion channel proteins. The two major forms are ROMANO-WARD SYNDROME and JERVELL-LANGE NIELSEN SYNDROME.
A type of cardiac arrhythmia with premature atrial contractions or beats caused by signals originating from ectopic atrial sites. The ectopic signals may or may not conduct to the HEART VENTRICLES. Atrial premature complexes are characterized by premature P waves on ECG which are different in configuration from the P waves generated by the normal pacemaker complex in the SINOATRIAL NODE.
The veins that return the oxygenated blood from the lungs to the left atrium of the heart.
A malignant form of polymorphic ventricular tachycardia that is characterized by HEART RATE between 200 and 250 beats per minute, and QRS complexes with changing amplitude and twisting of the points. The term also describes the syndrome of tachycardia with prolonged ventricular repolarization, long QT intervals exceeding 500 milliseconds or BRADYCARDIA. Torsades de pointes may be self-limited or may progress to VENTRICULAR FIBRILLATION.
An autosomal dominant defect of cardiac conduction that is characterized by an abnormal ST-segment in leads V1-V3 on the ELECTROCARDIOGRAM resembling a right BUNDLE-BRANCH BLOCK; high risk of VENTRICULAR TACHYCARDIA; or VENTRICULAR FIBRILLATION; SYNCOPAL EPISODE; and possible sudden death. This syndrome is linked to mutations of gene encoding the cardiac SODIUM CHANNEL alpha subunit.
The thoracolumbar division of the autonomic nervous system. Sympathetic preganglionic fibers originate in neurons of the intermediolateral column of the spinal cord and project to the paravertebral and prevertebral ganglia, which in turn project to target organs. The sympathetic nervous system mediates the body's response to stressful situations, i.e., the fight or flight reactions. It often acts reciprocally to the parasympathetic system.
A short vein that collects about two thirds of the venous blood from the MYOCARDIUM and drains into the RIGHT ATRIUM. Coronary sinus, normally located between the LEFT ATRIUM and LEFT VENTRICLE on the posterior surface of the heart, can serve as an anatomical reference for cardiac procedures.
Modified cardiac muscle fibers composing the terminal portion of the heart conduction system.
AMINO ALCOHOLS containing the propanolamine (NH2CH2CHOHCH2) group and its derivatives.
A class I anti-arrhythmic agent (one that interferes directly with the depolarization of the cardiac membrane and thus serves as a membrane-stabilizing agent) with a depressant action on the heart similar to that of guanidine. It also possesses some anticholinergic and local anesthetic properties.
A general class of ortho-dihydroxyphenylalkylamines derived from tyrosine.
A tetrameric calcium release channel in the SARCOPLASMIC RETICULUM membrane of SMOOTH MUSCLE CELLS, acting oppositely to SARCOPLASMIC RETICULUM CALCIUM-TRANSPORTING ATPASES. It is important in skeletal and cardiac excitation-contraction coupling and studied by using RYANODINE. Abnormalities are implicated in CARDIAC ARRHYTHMIAS and MUSCULAR DISEASES.
The amount of BLOOD pumped out of the HEART per beat, not to be confused with cardiac output (volume/time). It is calculated as the difference between the end-diastolic volume and the end-systolic volume.
Symptoms of cerebral hypoperfusion or autonomic overaction which develop while the subject is standing, but are relieved on recumbency. Types of this include NEUROCARDIOGENIC SYNCOPE; POSTURAL ORTHOSTATIC TACHYCARDIA SYNDROME; and neurogenic ORTHOSTATIC HYPOTENSION. (From Noseworthy, JH., Neurological Therapeutics Principles and Practice, 2007, p2575-2576)
The dilatation of the aortic wall behind each of the cusps of the aortic valve.
In screening and diagnostic tests, the probability that a person with a positive test is a true positive (i.e., has the disease), is referred to as the predictive value of a positive test; whereas, the predictive value of a negative test is the probability that the person with a negative test does not have the disease. Predictive value is related to the sensitivity and specificity of the test.
A type of automatic, not reentrant, ectopic ventricular rhythm with episodes lasting from a few seconds to a minute which usually occurs in patients with acute myocardial infarction or with DIGITALIS toxicity. The ventricular rate is faster than normal but slower than tachycardia, with an upper limit of 100 -120 beats per minute. Suppressive therapy is rarely necessary.
Computer-assisted processing of electric, ultrasonic, or electronic signals to interpret function and activity.
Developmental abnormalities involving structures of the heart. These defects are present at birth but may be discovered later in life.
Production of an image when x-rays strike a fluorescent screen.
A nucleoside that is composed of ADENINE and D-RIBOSE. Adenosine or adenosine derivatives play many important biological roles in addition to being components of DNA and RNA. Adenosine itself is a neurotransmitter.
The ENTERIC NERVOUS SYSTEM; PARASYMPATHETIC NERVOUS SYSTEM; and SYMPATHETIC NERVOUS SYSTEM taken together. Generally speaking, the autonomic nervous system regulates the internal environment during both peaceful activity and physical or emotional stress. Autonomic activity is controlled and integrated by the CENTRAL NERVOUS SYSTEM, especially the HYPOTHALAMUS and the SOLITARY NUCLEUS, which receive information relayed from VISCERAL AFFERENTS.
The position or attitude of the body.
The muscular structure separating the right and the left lower chambers (HEART VENTRICLES) of the heart. The ventricular septum consists of a very small membranous portion just beneath the AORTIC VALVE, and a large thick muscular portion consisting of three sections including the inlet septum, the trabecular septum, and the outlet septum.
A condition in which the LEFT VENTRICLE of the heart was functionally impaired. This condition usually leads to HEART FAILURE; MYOCARDIAL INFARCTION; and other cardiovascular complications. Diagnosis is made by measuring the diminished ejection fraction and a depressed level of motility of the left ventricular wall.
The muscle tissue of the HEART. It is composed of striated, involuntary muscle cells (MYOCYTES, CARDIAC) connected to form the contractile pump to generate blood flow.
An alkaloid, originally from Atropa belladonna, but found in other plants, mainly SOLANACEAE. Hyoscyamine is the 3(S)-endo isomer of atropine.
A significant drop in BLOOD PRESSURE after assuming a standing position. Orthostatic hypotension is a finding, and defined as a 20-mm Hg decrease in systolic pressure or a 10-mm Hg decrease in diastolic pressure 3 minutes after the person has risen from supine to standing. Symptoms generally include DIZZINESS, blurred vision, and SYNCOPE.
Abrupt changes in the membrane potential that sweep along the CELL MEMBRANE of excitable cells in response to excitation stimuli.
Glycosides from plants of the genus DIGITALIS. Some of these are useful as cardiotonic and anti-arrhythmia agents. Included also are semi-synthetic derivatives of the naturally occurring glycosides. The term has sometimes been used more broadly to include all CARDIAC GLYCOSIDES, but here is restricted to those related to Digitalis.
A form of CARDIAC MUSCLE disease, characterized by left and/or right ventricular hypertrophy (HYPERTROPHY, LEFT VENTRICULAR; HYPERTROPHY, RIGHT VENTRICULAR), frequent asymmetrical involvement of the HEART SEPTUM, and normal or reduced left ventricular volume. Risk factors include HYPERTENSION; AORTIC STENOSIS; and gene MUTATION; (FAMILIAL HYPERTROPHIC CARDIOMYOPATHY).
Pathologic processes that affect patients after a surgical procedure. They may or may not be related to the disease for which the surgery was done, and they may or may not be direct results of the surgery.
A combination of congenital heart defects consisting of four key features including VENTRICULAR SEPTAL DEFECTS; PULMONARY STENOSIS; RIGHT VENTRICULAR HYPERTROPHY; and a dextro-positioned AORTA. In this condition, blood from both ventricles (oxygen-rich and oxygen-poor) is pumped into the body often causing CYANOSIS.
The hemodynamic and electrophysiological action of the HEART ATRIA.
A beta-1 adrenergic antagonist that has been used in the emergency treatment of CARDIAC ARRYTHMIAS.
A voltage-gated sodium channel subtype that mediates the sodium ion PERMEABILITY of CARDIOMYOCYTES. Defects in the SCN5A gene, which codes for the alpha subunit of this sodium channel, are associated with a variety of CARDIAC DISEASES that result from loss of sodium channel function.
This structure includes the thin muscular atrial septum between the two HEART ATRIA, and the thick muscular ventricular septum between the two HEART VENTRICLES.
A beta-2 adrenergic agonist used in the treatment of ASTHMA and BRONCHIAL SPASM.
A characteristic symptom complex.
Injections made into a vein for therapeutic or experimental purposes.
Transmission of the readings of instruments to a remote location by means of wires, radio waves, or other means. (McGraw-Hill Dictionary of Scientific and Technical Terms, 4th ed)
Irregular HEART RATE caused by abnormal function of the SINOATRIAL NODE. It is characterized by a greater than 10% change between the maximum and the minimum sinus cycle length or 120 milliseconds.
A disorder of cardiac function caused by insufficient blood flow to the muscle tissue of the heart. The decreased blood flow may be due to narrowing of the coronary arteries (CORONARY ARTERY DISEASE), to obstruction by a thrombus (CORONARY THROMBOSIS), or less commonly, to diffuse narrowing of arterioles and other small vessels within the heart. Severe interruption of the blood supply to the myocardial tissue may result in necrosis of cardiac muscle (MYOCARDIAL INFARCTION).
A cardioselective beta-1 adrenergic blocker possessing properties and potency similar to PROPRANOLOL, but without a negative inotropic effect.
An ethanolamine derivative that is an adrenergic alpha-1 agonist. It is used as a vasoconstrictor agent in the treatment of HYPOTENSION.
A form of inherited long QT syndrome (or LQT7) that is characterized by a triad of potassium-sensitive periodic paralysis, VENTRICULAR ECTOPIC BEATS, and abnormal features such as short stature, low-set ears, and SCOLIOSIS. It results from mutations of KCNJ2 gene which encodes a channel protein (INWARD RECTIFIER POTASSIUM CHANNELS) that regulates resting membrane potential.
A non-selective beta-adrenergic antagonist with a long half-life, used in cardiovascular disease to treat arrhythmias, angina pectoris, and hypertension. Nadolol is also used for MIGRAINE DISORDERS and for tremor.
Controlled physical activity which is performed in order to allow assessment of physiological functions, particularly cardiovascular and pulmonary, but also aerobic capacity. Maximal (most intense) exercise is usually required but submaximal exercise is also used.
The active sympathomimetic hormone from the ADRENAL MEDULLA. It stimulates both the alpha- and beta- adrenergic systems, causes systemic VASOCONSTRICTION and gastrointestinal relaxation, stimulates the HEART, and dilates BRONCHI and cerebral vessels. It is used in ASTHMA and CARDIAC FAILURE and to delay absorption of local ANESTHETICS.
Electric conductors through which electric currents enter or leave a medium, whether it be an electrolytic solution, solid, molten mass, gas, or vacuum.
Visualization of the heart structure and cardiac blood flow for diagnostic evaluation or to guide cardiac procedures via techniques including ENDOSCOPY (cardiac endoscopy, sometimes refered to as cardioscopy), RADIONUCLIDE IMAGING; MAGNETIC RESONANCE IMAGING; TOMOGRAPHY; or ULTRASONOGRAPHY.
The continuous measurement of physiological processes, blood pressure, heart rate, renal output, reflexes, respiration, etc., in a patient or experimental animal; includes pharmacologic monitoring, the measurement of administered drugs or their metabolites in the blood, tissues, or urine.
Failure of equipment to perform to standard. The failure may be due to defects or improper use.
The 10th cranial nerve. The vagus is a mixed nerve which contains somatic afferents (from skin in back of the ear and the external auditory meatus), visceral afferents (from the pharynx, larynx, thorax, and abdomen), parasympathetic efferents (to the thorax and abdomen), and efferents to striated muscle (of the larynx and pharynx).
Forced expiratory effort against a closed GLOTTIS.
Use of electric potential or currents to elicit biological responses.
Surgery performed on the heart.
Disorders of the AUTONOMIC NERVOUS SYSTEM occurring as a primary condition. Manifestations can involve any or all body systems but commonly affect the BLOOD PRESSURE and HEART RATE.
Patient care procedures performed during the operation that are ancillary to the actual surgery. It includes monitoring, fluid therapy, medication, transfusion, anesthesia, radiography, and laboratory tests.
A localized bulging or dilatation in the muscle wall of a heart (MYOCARDIUM), usually in the LEFT VENTRICLE. Blood-filled aneurysms are dangerous because they may burst. Fibrous aneurysms interfere with the heart function through the loss of contractility. True aneurysm is bound by the vessel wall or cardiac wall. False aneurysms are HEMATOMA caused by myocardial rupture.
A response by the BARORECEPTORS to increased BLOOD PRESSURE. Increased pressure stretches BLOOD VESSELS which activates the baroreceptors in the vessel walls. The net response of the CENTRAL NERVOUS SYSTEM is a reduction of central sympathetic outflow. This reduces blood pressure both by decreasing peripheral VASCULAR RESISTANCE and by lowering CARDIAC OUTPUT. Because the baroreceptors are tonically active, the baroreflex can compensate rapidly for both increases and decreases in blood pressure.
A condition caused by dysfunctions related to the SINOATRIAL NODE including impulse generation (CARDIAC SINUS ARREST) and impulse conduction (SINOATRIAL EXIT BLOCK). It is characterized by persistent BRADYCARDIA, chronic ATRIAL FIBRILLATION, and failure to resume sinus rhythm following CARDIOVERSION. This syndrome can be congenital or acquired, particularly after surgical correction for heart defects.
Naturally occurring or experimentally induced animal diseases with pathological processes sufficiently similar to those of human diseases. They are used as study models for human diseases.
Expenditure of energy during PHYSICAL ACTIVITY. Intensity of exertion may be measured by rate of OXYGEN CONSUMPTION; HEAT produced, or HEART RATE. Perceived exertion, a psychological measure of exertion, is included.
Abnormally low BLOOD PRESSURE that can result in inadequate blood flow to the brain and other vital organs. Common symptom is DIZZINESS but greater negative impacts on the body occur when there is prolonged depravation of oxygen and nutrients.
The HEART and the BLOOD VESSELS by which BLOOD is pumped and circulated through the body.
A prediction of the probable outcome of a disease based on a individual's condition and the usual course of the disease as seen in similar situations.
Pathological conditions involving the HEART including its structural and functional abnormalities.
The hemodynamic and electrophysiological action of the left HEART VENTRICLE. Its measurement is an important aspect of the clinical evaluation of patients with heart disease to determine the effects of the disease on cardiac performance.
A local anesthetic and cardiac depressant used as an antiarrhythmia agent. Its actions are more intense and its effects more prolonged than those of PROCAINE but its duration of action is shorter than that of BUPIVACAINE or PRILOCAINE.
An infant during the first month after birth.
Binary classification measures to assess test results. Sensitivity or recall rate is the proportion of true positives. Specificity is the probability of correctly determining the absence of a condition. (From Last, Dictionary of Epidemiology, 2d ed)
Theoretical representations that simulate the behavior or activity of the cardiovascular system, processes, or phenomena; includes the use of mathematical equations, computers and other electronic equipment.
A condition in which the RIGHT VENTRICLE of the heart was functionally impaired. This condition usually leads to HEART FAILURE or MYOCARDIAL INFARCTION, and other cardiovascular complications. Diagnosis is made by measuring the diminished ejection fraction and a depressed level of motility of the right ventricular wall.
An imbalance between myocardial functional requirements and the capacity of the CORONARY VESSELS to supply sufficient blood flow. It is a form of MYOCARDIAL ISCHEMIA (insufficient blood supply to the heart muscle) caused by a decreased capacity of the coronary vessels.
The heart rate of the FETUS. The normal range at term is between 120 and 160 beats per minute.
Contractile activity of the MYOCARDIUM.
A paravertebral sympathetic ganglion formed by the fusion of the inferior cervical and first thoracic ganglia.
Drugs that selectively bind to and activate beta-adrenergic receptors.
The hemodynamic and electrophysiological action of the HEART VENTRICLES.
Striated muscle cells found in the heart. They are derived from cardiac myoblasts (MYOBLASTS, CARDIAC).
The relationship between the dose of an administered drug and the response of the organism to the drug.
A heterogeneous condition in which the heart is unable to pump out sufficient blood to meet the metabolic need of the body. Heart failure can be caused by structural defects, functional abnormalities (VENTRICULAR DYSFUNCTION), or a sudden overload beyond its capacity. Chronic heart failure is more common than acute heart failure which results from sudden insult to cardiac function, such as MYOCARDIAL INFARCTION.
An imprecise term which may refer to a sense of spatial disorientation, motion of the environment, or lightheadedness.
Electromagnetic waves with frequencies between about 3 kilohertz (very low frequency - VLF) and 300,000 megahertz (extremely high frequency - EHF). They are used in television and radio broadcasting, land and satellite communications systems, radionavigation, radiolocation, and DIATHERMY. The highest frequency radio waves are MICROWAVES.
Precursor of epinephrine that is secreted by the adrenal medulla and is a widespread central and autonomic neurotransmitter. Norepinephrine is the principal transmitter of most postganglionic sympathetic fibers and of the diffuse projection system in the brain arising from the locus ceruleus. It is also found in plants and is used pharmacologically as a sympathomimetic.
An aspect of personal behavior or lifestyle, environmental exposure, or inborn or inherited characteristic, which, on the basis of epidemiologic evidence, is known to be associated with a health-related condition considered important to prevent.
Serotonin derivative proposed as potentiator for hypnotics and sedatives.
Agents that have a strengthening effect on the heart or that can increase cardiac output. They may be CARDIAC GLYCOSIDES; SYMPATHOMIMETICS; or other drugs. They are used after MYOCARDIAL INFARCT; CARDIAC SURGICAL PROCEDURES; in SHOCK; or in congestive heart failure (HEART FAILURE).
The artificial substitution of heart and lung action as indicated for HEART ARREST resulting from electric shock, DROWNING, respiratory arrest, or other causes. The two major components of cardiopulmonary resuscitation are artificial ventilation (RESPIRATION, ARTIFICIAL) and closed-chest CARDIAC MASSAGE.
A nicotinic antagonist most commonly used as an experimental tool. It has been used as a ganglionic blocker in the treatment of hypertension but has largely been supplanted for that purpose by more specific drugs.
The qualitative or quantitative estimation of the likelihood of adverse effects that may result from exposure to specified health hazards or from the absence of beneficial influences. (Last, Dictionary of Epidemiology, 1988)
A selective adrenergic beta-1 blocking agent that is commonly used to treat ANGINA PECTORIS; HYPERTENSION; and CARDIAC ARRHYTHMIAS.
The long-term (minutes to hours) administration of a fluid into the vein through venipuncture, either by letting the fluid flow by gravity or by pumping it.
Diseases of the parasympathetic or sympathetic divisions of the AUTONOMIC NERVOUS SYSTEM; which has components located in the CENTRAL NERVOUS SYSTEM and PERIPHERAL NERVOUS SYSTEM. Autonomic dysfunction may be associated with HYPOTHALAMIC DISEASES; BRAIN STEM disorders; SPINAL CORD DISEASES; and PERIPHERAL NERVOUS SYSTEM DISEASES. Manifestations include impairments of vegetative functions including the maintenance of BLOOD PRESSURE; HEART RATE; pupil function; SWEATING; REPRODUCTIVE AND URINARY PHYSIOLOGY; and DIGESTION.
The process of generating three-dimensional images by electronic, photographic, or other methods. For example, three-dimensional images can be generated by assembling multiple tomographic images with the aid of a computer, while photographic 3-D images (HOLOGRAPHY) can be made by exposing film to the interference pattern created when two laser light sources shine on an object.
An abnormally low volume of blood circulating through the body. It may result in hypovolemic shock (see SHOCK).
A beta-adrenergic antagonist used in the treatment of hypertension, angina pectoris, arrhythmias, and anxiety.
Any operation on the spinal cord. (Stedman, 26th ed)
A state characterized by loss of feeling or sensation. This depression of nerve function is usually the result of pharmacologic action and is induced to allow performance of surgery or other painful procedures.
Nerves and plexuses of the autonomic nervous system. The central nervous system structures which regulate the autonomic nervous system are not included.
Drugs that bind to and block the activation of ADRENERGIC BETA-1 RECEPTORS.
Post-systolic relaxation of the HEART, especially the HEART VENTRICLES.
Studies to determine the advantages or disadvantages, practicability, or capability of accomplishing a projected plan, study, or project.
A guanidinium antihypertensive agent that acts by blocking adrenergic transmission. The precise mode of action is not clear.
Optical imaging techniques used for recording patterns of electrical activity in tissues by monitoring transmembrane potentials via FLUORESCENCE imaging with voltage-sensitive fluorescent dyes.
Drugs that mimic the effects of stimulating postganglionic adrenergic sympathetic nerves. Included here are drugs that directly stimulate adrenergic receptors and drugs that act indirectly by provoking the release of adrenergic transmitters.
The measurement of magnetic fields generated by electric currents from the heart. The measurement of these fields provides information which is complementary to that provided by ELECTROCARDIOGRAPHY.
A statistical technique that isolates and assesses the contributions of categorical independent variables to variation in the mean of a continuous dependent variable.
A congenital heart defect characterized by downward or apical displacement of the TRICUSPID VALVE, usually with the septal and posterior leaflets being attached to the wall of the RIGHT VENTRICLE. It is characterized by a huge RIGHT ATRIUM and a small and less effective right ventricle.

Site of myocardial infarction. A determinant of the cardiovascular changes induced in the cat by coronary occlusion. (1/1171)

The influence of site of acute myocardial infarction on heart rate, blood pressure, cardiac output, total peripheral resistance (TPR), cardiac rhythm, and mortality was determined in 58 anesthetized cats by occlusion of either the left anterior descending (LAD), left circumflex or right coronary artery. LAD occlusion resulted in immediate decrease in cardiac output, heart rate, and blood pressure, an increase in TPR, and cardiac rhythm changes including premature ventricular beats, ventricular tachycardia, and occasionally ventricular fibrillation. The decrease in cardiac output and increase in TPR persisted in the cats surviving a ventricular arrhythmia. In contrast, right coronary occlusion resulted in a considerably smaller decrease in cardiac output. TPR did not increase, atrioventricular condition disturbances were common, and sinus bradycardia and hypotension persisted in the cats recovering from an arrhythmia. Left circumflex ligation resulted in cardiovascular changes intermediate between those produced by occlusion of the LAD or the right coronary artery. Mortality was similar in each of the three groups. We studied the coronary artery anatomy in 12 cats. In 10, the blood supply to the sinus node was from the right coronary artery and in 2, from the left circumflex coronary artery. The atrioventricular node artery arose from the right in 9 cats, and from the left circumflex in 3. The right coronary artery was dominant in 9 cats and the left in 3. In conclusion, the site of experimental coronary occlusion in cats is a major determinant of the hemodynamic and cardiac rhythm changes occurring after acute myocardial infarction. The cardiovascular responses evoked by ligation are related in part to the anatomical distribution of the occluded artery.  (+info)

Regional differences in the recovery course of tachycardia-induced changes of atrial electrophysiological properties. (2/1171)

BACKGROUND: Regional differences in recovery of tachycardia-induced changes of atrial electrophysiological properties have not been well studied. METHODS AND RESULTS: In the control group (5 dogs), atrial effective refractory period (AERP) and inducibility of atrial fibrillation (AF) were assessed before and every 4 hours for 48 hours after complete atrioventricular junction (AVJ) ablation with 8-week VVI pacing. In experimental group 1 (15 dogs), AERP and inducibility of AF were assessed before and after complete AVJ ablation with 8-week rapid right atrial (RA) pacing (780 bpm) and VVI pacing. In experimental group 2 (7 dogs), AERP and inducibility of AF were assessed before and after 8-week rapid left atrial (LA) pacing and VVI pacing. AERP and inducibility and duration of AF were obtained from 7 epicardial sites. In the control group, atrial electrophysiological properties obtained immediately and during 48-hour measurements after pacing did not show any change. In the 2 experimental groups, recovery of atrial electrophysiological properties included a progressive recovery of AERP shortening, recovery of AERP maladaptation, and decrease of duration and episodes of reinduced AF. However, recovery of shortening and maladaptation of AERP and inducibility of AF was slower at the LA than at the RA and Bachmann's bundle. CONCLUSIONS: The LA had a slower recovery of tachycardia-induced changes of atrial electrophysiological properties, and this might play a critical role in initiation of AF.  (+info)

Ketotifen and cardiovascular effects of xamoterol following single and chronic dosing in healthy volunteers. (3/1171)

AIMS: To study whether desensitization occurs after long-term administration of the 1-adrenoceptor partial agonist xamoterol and, if so, whether this can be influenced by ketotifen. METHODS: In a double-blind, randomized design 10 young, healthy males received ketotifen (2 x 1 mg day(-1) p.o.) or placebo for 3 weeks with xamoterol (2 x 200 mg day(-1) p.o.) administered concomitantly during the last 2 weeks. 'l1-adrenoceptor mediated responses were assessed as exercise-induced tachycardia and isoprenaline-induced shortening of heart rate corrected electromechanical systole (QS2c); isoprenaline-induced tachycardia was measured as a mixed beta1-/beta2-adrenoceptor-mediated effect. RESULTS: The first dose of xamoterol significantly increased resting heart rate and systolic blood pressure and significantly shortened QS2c. The last dose of xamoterol after 2 weeks of treatment still produced the same responses. Ketotifen did not influence these effects of xamoterol on resting haemodynamics. The first dose of xamoterol caused a rightward shift of the exercise- and isoprenaline-induced tachycardia (mean dose ratios+/-s.e.mean: 1.20+/-0.05 and 2.46+/-0.23) and the isoprenaline-evoked shortening of QS2c (dose ratio 3.59+/-0.68). This rightward shift was even more pronounced after 2 weeks xamoterol treatment. This additional rightward shift after 2 weeks of xamoterol was not affected by ketotifen (mean difference (95% CI) of log transformed dose ratios between placebo and ketotifen: exercise tachycardia 0.001 (-0.03; 0.04); isoprenaline tachycardia 0.03 (-0.15; 0.21); isoprenaline induced shortening of QS2c 0.13 (-0.22; 0.48)). CONCLUSIONS: In humans xamoterol is a partial beta1-adrenoceptor agonist with positive chrono- and inotropic effects at rest and antagonistic properties under conditions of beta-adrenoceptor stimulation. These effects were well maintained after chronic dosing with no signs of beta1-adrenoceptor desensitization. Ketotifen does not change the beta-adrenoceptor mediated responses of xamoterol after chronic dosing.  (+info)

LocaLisa: new technique for real-time 3-dimensional localization of regular intracardiac electrodes. (4/1171)

BACKGROUND: Estimation of the 3-dimensional (3D) position of ablation electrodes from fluoroscopic images is inadequate if a systematic lesion pattern is required in the treatment of complex arrhythmogenic substrates. METHODS AND RESULTS: We developed a new technique for online 3D localization of intracardiac electrodes. Regular catheter electrodes are used as sensors for a high-frequency transthoracic electrical field, which is applied via standard skin electrodes. We investigated localization accuracy within the right atrium, right ventricle, and left ventricle by comparing measured and true interelectrode distances of a decapolar catheter. Long-term stability was analyzed by localization of the most proximal His bundle before and after slow pathway ablation. Electrogram recordings were unaffected by the applied electrical field. Localization data from 3 catheter positions, widely distributed within the right atrium, right ventricle, or left ventricle, were analyzed in 10 patients per group. The relationship between measured and true electrode positions was highly linear, with an average correlation coefficient of 0.996, 0.997, and 0.999 for the right atrium, right ventricle, and left ventricle, respectively. Localization accuracy was better than 2 mm, with an additional scaling error of 8% to 14%. After 2 hours, localization of the proximal His bundle was reproducible within 1.4+/-1.1 mm. CONCLUSIONS: This new technique enables accurate and reproducible real-time localization of electrode positions in cardiac mapping and ablation procedures. Its application does not distort the quality of electrograms and can be applied to any electrode catheter.  (+info)

Effects of pacing-induced and balloon coronary occlusion ischemia on left atrial function in patients with coronary artery disease. (5/1171)

OBJECTIVES: The aim of this study was to compare left atrial (LA) function in 16 patients with distal left anterior descending (LAD) and in 16 patients with proximal left circumflex (LCx) coronary artery stenosis at rest and immediately after pacing-induced tachycardia (LAD-pacing [P] and LCx-P) or coronary occlusion (LAD-CO and LCx-CO). BACKGROUND: During left ventricular (LV) ischemia, compensatory augmentation of LA contraction enhances LV filling and performance. The left atrium is supplied predominantly by branches arising from the LCx. Therefore, we hypothesized that one mechanism for the loss of atrial contraction may be ischemic LA dysfunction. METHODS: Left ventricular and LA pressure-area relations were derived from simultaneous double-tip micromanometer pressure recordings and automatic boundary detection echocardiograms. RESULTS: Immediately after pacing or after coronary occlusion, LV end-diastolic pressure, LV relaxation, LA mean pressure and LV stiffness significantly increased in all patients. However, the area of the A loop of the LA pressure-area relation, representing the LA pump function, significantly decreased in groups LCx-P and LCx-CO (from 14+/-3 to 9+/-2, and from 16+/-4 to 9+/-2 mm Hg.cm2, respectively, p < 0.05), whereas it increased in groups LAD-P and LAD-CO (from 12+/-3 to 54+/-10, and from 16+/-3 to 49+/-8 mm Hg.cm2, respectively, p < 0.001). CONCLUSIONS: In patients with LAD stenosis, LV supply or demand ischemia is associated with enhanced LA pump function. However, in patients with proximal LCx stenosis who develop the same type and degree of ischemia, LA branches might have been affected, rendering the LA ischemic and unable to increase its booster pump function.  (+info)

Frequency of arrhythmias and other cardiac abnormalities in fulminant hepatic failure. (6/1171)

In a series of 106 patients with fulminant hepatic failure and grade 4 encephalopathy, cardiac arrhythmias and other abnormalities occurred in 92 per cent. The most common was sinus tachycardia (75%) and this was the only abnormality in 22 per cent of the patients. Sudden cardiac arrest occurred in 25 per cent, various ectopic beats in 20 per cent, and heart block or bradycardia in 18 per cent. Other electrocardiographic abnormalities, mostly of the T wave and ST segment, were found in 31 per cent. Cardiac and respiratory arrests were usually unrelated to each other and both frequently occurred without warning. Only 7 out of 71 patients with arrhythmias other than sinus tachycardia survived, compared with 15 out of 31 patients without them (P less than 0-005). During the latter part of the series when an arrhythmia computer was used to monitor 38 patients, it was shown that significantly lower arterial oxygen levels occurred in those with arrhythmias, other than sinus tachycardia, than in those without. They were also found to be more acidotic and hyperkalaemic, and a higher number required dialysis and ventilation. Macroscopical cardiac abnormalities including scattered petechial haemorrhages, small pericardial effusions, and fatty, pale, and flabby ventricles, were found at necropsy in 64 per cent of the patients examined. Combinations of these macroscopical abnormalities occurred, particularly in the paracetamol overdose group. Another necropsy finding of possible significance in the pathogenesis of arrhythmias was cerebral oedema, present in 48 per cent of the patients examined, and often associated with coning of the brain stem. However, 7 of the 16 patients who suffered asystolic cardiac arrests had no macroscopical abnormality of either heart or brain. In the management of patients with fulminant hepatic failure continuous cardiac monitoring is essential. Correction of the biochemical and coagulation defects may decrease the frequency of arrhythmias but studies of the mechanism and control of cerebral oedema and its relation to cardiovascular function are urgently needed.  (+info)

Sino-aortic denervation augments the increase in blood pressure seen during paradoxical sleep in the rat. (7/1171)

Using a computer assisted telemetric system, we have re-examined the effect of sino-aortic denervation (SAD) on the changes in arterial blood pressure (AP) and heart rate (HR) during sleep in the rat suitably recovered from the operation. Eight 1 hourly polygraphic recordings were performed 4 weeks after the initial SAD surgery. In the SAD rats, the increase in AP during paradoxical sleep (PS) was much larger than that in sham-operated rats. HR in the SAD rats increased on-going from slow-wave sleep to PS, but it showed no change in sham-operated rats. The present study suggests that chronic SAD causes the enhanced AP increase during PS concomitantly with the persistent hypertension and tachycardia across sleep-wake states.  (+info)

Aryl propanolamines: comparison of activity at human beta3 receptors, rat beta3 receptors and rat atrial receptors mediating tachycardia. (8/1171)

1. The in vitro activity of four aryl propanolamines was compared to two prototypic beta3 receptor agonists, CGP 12177 and CL316243 at the human beta3 receptor, the rat beta3 receptor in the stomach fundus and receptors mediating atrial tachycardia. 2. L-739,574 was the most potent (EC50 = 9 nM) and selective agonist at the human beta3 receptor with high maximal response (74% of the maximal response to isoproterenol). 3. A phenol-biaryl ether analogue possessed modest affinity for the human beta3 receptor (EC50 = 246 nM), but was highly efficacious with a maximal response 82% of the maximal response to isoproterenol. The other derivatives were intermediate in potency with low maximal responses. 4. These agonists at the human beta3 receptor did not activate the rat beta3 receptor in the rat stomach fundus. In fact, the aryl propanolamines (10(-6) M) inhibited CL316243-induced activation of the rat beta3 receptor. Thus, agonist activity at the human beta3 receptor translated into antagonist activity at the rat beta3 receptor. 5. L739,574 and the phenol biaryl ether increased heart rate via beta1 receptors. 6. Although CGP12177 produced atrial tachycardia, neither the indole sulphonamide nor biphenyl biaryl ether did, although both had high affinity for the human beta3 receptor. Thus, the atrial tachycardic receptor was not identical to the human beta3 receptor. 7. These studies (a) characterized four aryl propanolamines with high affinity at the human beta3 receptor, (b) found that they were antagonists at the rat beta3 receptor, an observation with profound implications for in vivo rat data, and (c) established that the rodent atrial non-beta1, beta2 or beta3 tachycardic receptor was also unrelated to the human beta3 receptor.  (+info)

Tachycardia, also called tachyarrhythmia, is a heart rate that exceeds the normal resting rate. In general, a resting heart rate over 100 beats per minute is accepted as tachycardia in adults. Heart rates above the resting rate may be normal (such as with exercise) or abnormal (such as with electrical problems within the heart). The upper threshold of a normal human resting heart rate is based on age. Cutoff values for tachycardia in different age groups are fairly well standardized; typical cutoffs are listed below: 1-2 days: Tachycardia > 159 beats per minute (bpm) 3-6 days: Tachycardia >166 bpm 1-3 weeks: Tachycardia >182 bpm 1-2 months: Tachycardia >179 bpm 3-5 months: Tachycardia >186 bpm 6-11 months: Tachycardia >169 bpm 1-2 years: Tachycardia >151 bpm 3-4 years: Tachycardia >137 bpm 5-7 years: Tachycardia >133 bpm 8-11 years: Tachycardia >130 bpm 12-15 years: Tachycardia >119 bpm >15 years - adult: Tachycardia >100 bpm Heart rate is considered in the context of the prevailing clinical ...
Mechanistic studies of human heart failure are complicated by the prolonged time course of development of the disease, the technical challenges of isolating cardiac tissue or cells from explanted hearts, the inability to investigate the early time course of cellular alterations, and the lack of control over experimental conditions. Thus, it is fortunate that the canine tachycardia-induced heart failure model so closely reproduces the known hemodynamic and ionic changes that have been identified in human hearts. The present findings indicate that, in addition to the electrophysiological changes noted in earlier studies, significant alterations in Ca2+ handling occur in isolated myocytes from failing hearts, following the general pattern of human studies. Through biochemical and functional measurements in the same hearts, we have found strong evidence in support of the hypothesis that the induction of heart failure triggers a shift in the balance of cytosolic Ca2+ extrusion mechanisms from SR Ca2+ ...
Successful Ablation for Atrial Tachycardia Originated from Sinus Venosa with Tachycardia-Induced Cardiomyopathy. . Biblioteca virtual para leer y descargar libros, documentos, trabajos y tesis universitarias en PDF. Material universiario, documentación y tareas realizadas por universitarios en nuestra biblioteca. Para descargar gratis y para leer online.
Fetal Tachycardia (FT) is described as increase in baseline fetal heart rate (FHR) above 160bpm. Mild fetal tachycardia is described as 161-180bpm and severe tachycardia is defined as greater than 180bpm for at least three minutes. The fetal tachycardia causes include maternal fever, dehydration or anxiety, maternal ketosis, medications like anticholinergic medications, sympathomimetic medications like terbutaline, fetal movement, preterm fetus, maternal thyrotoxicosis and maternal anaemia1. Fetal tachycardia is considered significant (any range ,160-180bpm) in the presence of maternal pyrexia as Chorioamnionitis is suspected. Fetal arrhythmia or congenital defect is associated with FHR more than 200 bpm. Baseline FHR tachycardia represents an increase in sympathetic and or a decrease in parasympathetic autonomic nervous system tone1.. Complicated fetal tachycardia in the presence of decelerations or maternal fever qualify the decision for delivering the baby in view of fetal distress and ...
Tachycardia refers to a condition of an irregularly rapid beating of the heart. Tachycardia is defined as a heart rate greater than 100 beats per minute, while a normal heart beats is measured at 72 beats per minute. Tachycardia occurs when the electrical impulses that coordinate heartbeats do not function properly and cause the heart to beat irregularly fast. In some situations, an increased heart rate can be normal, such as during exercise and stress. However, depending on the extent of the Tachycardia and the overall health of the patient, tachycardia may be dangerous and require serious medical attention.. If the heart rate is abnormally increased for an extended period of time, oxygen and carbon dioxide levels in the bloodstream can be dramatically altered. Tachycardia can cause stress on the heart because the heart muscle is forced to take less time to relax between each contraction. Patients with Tachycardia may often feel out of breath because the faster the heart beats, the more oxygen ...
TY - JOUR. T1 - Tachycardia in Post-Infarction Hearts. T2 - Insights from 3D Image-Based Ventricular Models. AU - Arevalo, Hermenegild. AU - Plank, Gernot. AU - Helm, Patrick. AU - Halperin, Henry R. AU - Trayanova, Natalia. PY - 2013/7/2. Y1 - 2013/7/2. N2 - Ventricular tachycardia, a life-threatening regular and repetitive fast heart rhythm, frequently occurs in the setting of myocardial infarction. Recently, the peri-infarct zones surrounding the necrotic scar (termed gray zones) have been shown to correlate with ventricular tachycardia inducibility. However, it remains unknown how the latter is determined by gray zone distribution and size. The goal of this study is to examine how tachycardia circuits are maintained in the infarcted heart and to explore the relationship between the tachycardia organizing centers and the infarct gray zone size and degree of heterogeneity. To achieve the goals of the study, we employ a sophisticated high-resolution electrophysiological model of the infarcted ...
A pacing system provided with a mode switching feature and ventricular rate regularization (VRR) function adapted to stabilize or regularize ventricular heart rate during chronic or paroxysmal atrial tachyarrhythmia. In a preferred embodiment, the pacing system nominally operates in an atrial synchronized pacing mode such as DDD or DDDR pacing mode. In response to detection of atrial rhythm characteristics consistent with an atrial tachyarrhythmia, e.g., atrial fibrillation, a mode switch into a non-atrial synchronized, ventricular rate regularization pacing mode, e.g. DDIR or VDIR pacing mode, is made. If the VRR function is programmed on, the ventricular pacing rate based upon a rate responsive sensor derived ventricular pacing rate modulated on a beat by beat basis by preceding intrinsic or paced ventricular events, the stability of the intrinsic ventricular heart rate, and any atrial pace events to regularize the ventricular pacing rate. The pacing system may also be permanently programmed to the
The freeMD virtual doctor has found 32 conditions that can cause Abdominal Tenderness and Tachycardia. There are 6 common conditions that can cause Abdominal Tenderness and Tachycardia. There are 4 somewhat common conditions that can cause Abdominal Tenderness and Tachycardia. There are 7 uncommon conditions that can cause Abdominal Tenderness and Tachycardia. There are 15 rare conditions that can cause Abdominal Tenderness and Tachycardia.
This page includes the following topics and synonyms: Tachycardia due to Medications, Drug Induced Tachycardia, Toxin Induced Tachycardia.
The study of conversion of AF to sinus rhythm provides information on changes associated with the resumption of atrial contraction and AV synchrony leading to a regularized, rate-controlled ventricular response. Previous studies have documented improvement in cardiac output ([75]) and cardiopulmonary exercise testing variables of ventricular rate control, maximal oxygen uptake and anaerobic threshold ([76, 77]) after cardioversion to sinus rhythm. More recently, a small, prospective study of patients with a diagnosis of idiopathic dilated cardiomyopathy and chronic AF reported a significant increase in left ventricular ejection fraction after pharmacologic or electrical cardioversion to sinus rhythm ([78]). Whether improvement in cardiac function was due to restoration of atrial systolic function and AV synchrony versus reversal of an underlying cardiomyopathy associated with rapid and irregular ventricular rates during AF was not resolved by this study. Subsequent study of atrial and ...
The best 16 synonyms for tachycardia, including: hypotension, tachypnoea, dysrhythmias, bronchospasm, palpitation, dyspnoea, stridor, low blood pressure, hypoxaemia, ventricular-fibrillation, oedema and more... Find another word for tachycardia at YourDictionary.
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The rhythm is a regular WCT ( = Wide-Complex Tachycardia) at ~ 210/minute without sign of atrial activity. Dr. Smith has reviewed the principal differential diagnosis. I dont think QRS duration is particularly helpful here, given that the patient is a 12-year old child (for whom normal QRS duration is a bit less than in adults). So the QRS is definitely wide, though not excessively so - but probably overlaps most entities to be considered. What strikes me - is the obvious suggestion of bifascicular block (RBBB/LAHB), though of atypical morphology. In an otherwise healthy child (who does not have congenital heart disease or other structural heart abnormality) - one would expect a more typical RBBB morphology with a clear triphasic appearance with taller right-rabbit ear in lead V1, instead of the atypical picture we see here in V1,V2,V3. Considering this and integrating it with the important Learning Points Dr. Smith emphasizes (ie, that fascicular VT often occurs in otherwise normal hearts) - ...
Most of us (assuming no pediatric cardiologists are listening) do not see a large number of pediatric arrhythmias (certainly far less than weve all seen in adults). As a result - level of comfort in both diagnosis and management tends to be less than that attained with adult arrhythmias. As wonderfully emphasized by Dr. Smith by the 2 pediatric arrhythmia examples shown here - accurate determination of rate is CRITICAL in the assessment of pediatric arrhythmias. For this determination - I have found use of the Every-other-Beat (or sometimes, Every-third-Beat) method a user-friendly way to rapidly and accurate determine heart rate for any regular fast rhythm. While most of us recall, 300- 150 - 100 - 75 - 60 - 50 as the answers for rate calculation when the R-R interval of a regular tachycardia is 1, 2, 3, 4, 5 or 6 large boxes, respectively - this becomes problematic with very fast rates. Thats because small differences in measurements may translate into significant under- or ...
Improved methods and devices perform tachycardia detection and anti-tachycardia pacing (ATP) to convert a tachycardia (e.g., VT or AT) to normal sinus rhythm. According to one embodiment, an anti-tachycardia pacing method includes sensing, during sinus rhythm, first and second cardiac signals at first and second sites, respectively, in a patients heart. The first and second sites include left and right ventricles or left and right atria, for example. The method further includes sensing third and fourth cardiac signals at the first and second sites, respectively, during a tachycardia (e.g., ventricular tachycardia or atrial tachycardia). The cardiac signals are processed to provide respective values. One or more anti-tachycardia pacing pulses are delivered at the site closest to the reentrant circuit based on a comparison of a first ratio of the first and third values and a second ratio of the second and fourth values. Unipolar sensing of the cardiac signals may be employed by, for example,
Tachycardia: Abnormally rapid heartbeat, usually with a HEART RATE above 100 beats per minute for adults. Tachycardia accompanied by disturbance in the cardiac depolarization (cardiac arrhythmia) is called tachyarrhythmia.
TITLE PAGETitle: Interesting changeover from short VA to long VA tachycardia with single atrial premature depolarisation: What is the mechanism?Short title: Concomitant AVNRT and atrial tachycardia: Killing two birds with one stone.
What is tachycardia arrhythmia? Learn about tachycardia arrhythmia (fast heartbeat), including causes, risk factors, symptoms, diagnosis and treatment from the cardiology experts at Mercy Health.
Fascinating case with a number of interesting insights to learn. Diagnosis of alternate preexcitation on the last tracing is subtle, but important to recognize. The 2 other thoughts I considered in my differential diagnosis for this last ECG were: i) electrical alternans; and ii) ventricular bigeminy with fusion from end-diastolic PVCs - but neither fit (thats because we see DELTA waves in several leads on this tracing that provide the diagnosis). Otherwise - the T wave peaking we see on the tachycardia ECGs is extremely interesting. I have often seen T wave peaking with tachycardia (and on stress tests) in otherwise healthy young adults - but I dont expect ST elevation. In addition, this patient was apparently markedly dehydrated on each occasion when the ECG was done. Hyperkalemia (perhaps secondary to volume contraction) not uncommonly leads to T wave peaking with recurrent episodes, but again supposedly not associated with ST elevation (Wonder what serum K+ values were in this patient ...
Tachycardia refers to a fast resting heart rate, usually over 100 beats per minute. Tachycardia can be dangerous, depending on its underlying cause and on
I. Problem/Condition. Tachycardias include all heart rhythms with a rate , 100 bpm. They can be divided into two primary categories: ) Narrow Complex Tachycardias (NCT) which have a QRS duration , 120 msec ) Wide Complex Tachycardias (WCT) which have a QRS duration , 120 msec The NCTs are due to rapid activation of…. ...
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BACKGROUND: Catheter ablation (CA) therapy is the first choice treatment in adults with heart rhythm disturbances. The arrhythmias in adults are mainly conditioned by coronary disease. Etiology of arrhythmias in children is mostly associated with inherited heart disorders. According to the current guidelines the CA is widely used in children, indicating the need to make it more achievable in pediatric population. AIM: To assess efficacy and safety of CA in children with different types of arrhythmias on the initial learning curve at newly built Ablation Center in The Independent Pediatric Hospital of Medical University of Warsaw ...
Most arrhythmia including Tachycardia are not serious However, any abnormality associated with the heart can quickly develop into a serious, or even life
An interesting technicality in the use of the PERC rule to rule out pulmonary embolism is the tachycardia component - it asks not whether the patient is tachycardic at the time of the application of the rule, or whether tachycardia was sustained throughout the emergency department stay, but instead whether the patient had (as described by Jeff Kline in his great review article on PE diagnosis and risk stratification): 3. Pulse ,100 beats/min during entire stay in ED. Meaning, even transient tachycardia may suggest a life-threatening diagnosis, even if it resolves while the patient is in the emergency department, and were probably PERCing out a whole bunch of patients inappropriately, at least according to Kline (who, notably, testifies a whole bunch as an expert witness in cases of missed pulmonary emboli).. I recently had a handful of patients in whom concerning blood pressures were measured and documented, which then resolved when vital signs were re-checked or after a small quantity of ...
You are working one evening in the emergency department when a 60-something year old female is slotted for a room. Her chief complaint? Fever, weakness, vomiting. Seeing that her triage heart rate was 157, you leave your granola bar where it is and immediately walk into the room to assess her. You see an elderly-appearing female in moderate respiratory distress. Her temperature is 38.2, blood pressure is 125/87, RR is 32, oxygen saturation is 93% on 5L NC. She has a history of a bone-marrow transplant and is chronically immunosuppressed. She endorses poor PO intake and several episodes of emesis over the last few days. She says that she came in today when she developed some shortness of breath as well. She denies any chest pain or palpitations. On exam, her mucous membranes are dry, her abdomen non-tender, and her breath sounds are decreased in the right base. You are a bit disturbed by the looks of her rhythm strip on the monitor so you get a 12-lead EKG ...
A Computational Study of the Effects of Tachycardia-Induced Remodeling on Calcium Wave Propagation in Rabbit Atrial Myocytes: In atrial cardiomyocytes without a
Learn more about Tachycardia at Reston Hospital Center DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...
Learn more about Tachycardia at Grand Strand Medical Center DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...
Tachycardia is a condition where a persons heart is beating abnormally fast. When we become more active our heart rate increases to meet the increased
Atrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics (costs for program #250319) ✔ University Hospital Halle (Saale) ✔ Department of Cardiology and Angiology (Department of Internal Medicine III) ✔ BookingHealth.com
Atrioventricular nodal Rreentrant tachycardia (AVNRT) diagnostics (costs for program #144277) ✔ Academic Hospital Neuperlach ✔ Department of Cardiology, Pneumology and Internal Intensive Care ✔ BookingHealth.com
Tachycardia is a rapid heart rate caused by a problem in the hearts electrical system. Find out more about this common heart rhythm disorder (arrhythmia).
A pacemaker-mediated tachycardia (PMT) can be defined as any condition in which a pacemaker paces the ventricles at rates that are inappropriately fast. This can be due to (1) a rate response setting that is too sensitive, (2) tracking of atrial noise (such as what may occur with electromagnetic interference), (3) inappropriate pacemaker mani...
A heart rate of more than 100 beats per minute (BPM) in adults is called tachycardia. Whats too fast for you may depend on your age and physical condition.
Rapid and abnormal increase in heart rate more than 100 beats a minute in rest is tachycardia. It is caused by abnormal electrical impulses originated in the heart chambers with a physiological or pathological background.
Its been a while since I been on here, but I must say life is good, I am truly blessed. My pvcs are almost completely gone, I may feel only a couple a week and I havent had a tachycardia attack in over three months, I must say again God is good. I was once afraid to live on my own because if my heart issues, but I recently moved into my own house three weeks ago. I posted a while back about the doctor putting me on Mirtazapine for anxiety and weight gain, it has really helped me alot, I started off at 111lbs, I am now 134lbs ...
Question - Suffering from SVT tachycardia. Prescribed with metroprolol. Is it safe to go for skiing?. Ask a Doctor about diagnosis, treatment and medication for Increased heart rate, Ask a Cardiologist
how high can an axiety attact drive ones HR up? to 110, 120, 130?? I fhigh, one will confuse the adrenaline caused high HR that stops in a few minutes with tachycardia. More and more I believe, aft...
i did a post on here regarding tachycardia warnings well this is part 2 after suffering complications in hospital which resulted in very low potassium it would seem that i am back to square one...
Dont just change employers, change your life with Healthworks, Inc. This webinar focuses on the core concepts of a basic electrophysiology study and tools to induce tachycardia.
A disruption in the normal electrical impulses controlling the pumping action of the heart causes tachycardia, according to Mayo Clinic. Many different factors cause these disruptions and the related...
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Had cathetor ablation following a 189 heart beat which I could not control (prior to that over a number of years had, say 120 beats - I could stop by holding my breath a few times and it would disappe
I have been experiencing a fast, pounding heart rate for over a month now. It gets worse when I stand up. I get light headed, very short of breath, dizzy, weak, and feel as if Im going to faint. I wen...
I have been wearing a chest hr monitor for the last week and have been concerned that I seem to be tachycardiac in the afternoons. My resting heart...
DI-fusion, le Dépôt institutionnel numérique de lULB, est loutil de référencementde la production scientifique de lULB.Linterface de recherche DI-fusion permet de consulter les publications des chercheurs de lULB et les thèses qui y ont été défendues.
TY - JOUR. T1 - Chronic orthostatic intolerance. T2 - A disorder with discordant cardiac and vascular sympathetic control. AU - Furlan, Raffaello. AU - Jacob, Giris. AU - Snell, Marie. AU - Robertson, David. AU - Porta, Alberto. AU - Harris, Paul. AU - Mosqueda-Garcia, Rogelio. PY - 1998/11/17. Y1 - 1998/11/17. N2 - Background - Chronic orthostatic intolerance (COI) is a debilitating autonomic condition in young adults. Its neurohumoral and hemodynamic profiles suggest possible alterations of postural sympathetic function and of baroreflex control of heart rate (HR). Methods and Results - In 16 COI patients and 16 healthy volunteers, intra-arterial blood pressure (BP), ECG, central venous pressure (CVP), and muscle sympathetic nerve activity (MSNA) were recorded at rest and during 75°tilt. Spectral analysis of RR interval and systolic arterial pressure (SAP) variabilities provided indices of sympathovagal modulation of the sinoatrial node (ratio of low-frequency to high-frequency components, ...
Objectives. The purpose of this study was to determine the predictors of electrically induced ventricular tachycardia in a large sample of patients with unexplained syncope and to examine the value of the signal-averaged electrocardiogram (ECG) in those patient subsets with varying pretest probability of ventricular tachycardia.. Background. In patients with unexplained syncope, electrophysiologic study can provide important diagnostic information, such as inducibility of ventricular tachycardia. The signal-averaged ECG can predict inducible ventricular tachycardia, but its utility has not been prospectively studied in a large group of patients with unexplained syncope.. Methods. At six hospitals, 189 consecutive patients with unexplained syncope underwent signal-averaged ECG and electrophysiologic studies.. Results. Ventricular tachycardia was induced in 28 patients (15%). Univariate predictors of ventricular tachycardia included history of previous myocardial infarction, reduced left ...
There are several ways to classify ventricular tachycardia, such as morphology or based on the duration of the episodes. From a morphological point of view, ventricular tachycardia can be divided into monomorphic ventricular tachycardia, scar related monomorphic ventricular tachycardia, polymorphic ventricular tachycardia and RVOT tachycardia. The second type of tachycardia mentioned here is the most common, especially with patients who have already suffered from a heart attack and it can also lead to death. Based on the duration, ventricular tachycardia can be divided into two types: non-sustained ventricular tachycardia if the fast rhythm ends itself within thirty seconds and sustained ventricular tachycardia if the rhythm lasts longer than thirty seconds.. Determining what causes is can be very complicated and requires the input of a specialized doctor and several tests, the most common of which is effectuated with the help of an EKG. The manifestations of ventricular tachycardia can be very ...
This condition can cause dizziness when moving to an upright position and can be triggered by infections. They found that older people, women, and those who had five or more symptoms in the first week of becoming ill with Covid-19 were more likely to develop long Covid. Researchers have analysed data from the COVID Symptom Study app to discover who is most at risk of developing long Covid. The BHF is supporting the following research projects: The BHF is supporting the following research projects: The BHF is supporting the following research projects: The most common symptoms are feeling lightheaded, palpitations (being aware of your heartbeat) and fatigue. The most common symptoms are feeling lightheaded, palpitations (being aware of your heartbeat) and fatigue. There is emerging evidence that a few people who develop long Covid have similar symptoms to people with a condition known as postural tachycardia syndrome, or postural orthostatic tachycardia syndrome (PoTS).. ...
The Postural Tachycardia Syndrome (POTS) is the most common disorder seen in autonomic clinics. Cardinal hemodynamic feature of this chronic and debilitating disorder of orthostatic tolerance is an exaggerated orthostatic tachycardia (≥30 bpm increase in HR with standing) in the absence of orthostatic hypotension. There are multiple pathophysiological mechanisms that underlie POTS. Some patients with POTS have evidence of elevated sympathoneural tone. This hyperadrenergic state is likely a driver of the excessive orthostatic tachycardia. Another common pathophysiological mechanism in POTS is a hypovolemic state. Many POTS patients with a hypovolemic state have been found to have a perturbed renin-angiotensin-aldosterone profile. These include inappropriately low plasma renin activity and aldosterone levels with resultant inadequate renal sodium retention. Some POTS patients have also been found to have elevated plasma angiotensin II (Ang-II) levels, with some studies suggesting problems with
Radiofrequency ablation of ventricular tachycardia is now well established in the management of patients with focal ventricular tachycardia occurring without structural heart disease.1 ,2 ,11 Although high success rates have been reported in this context and much has been learnt about mapping and ablation, they form a small proportion of the total population of patients with ventricular tachycardia.8 ,9 Ischaemic heart disease is the dominant cause of ventricular tachycardia, and RF ablation in this setting is beset with problems. The traditional approach has been to induce ventricular tachycardia and then map the induced rhythm and perform ablation during the tachycardia, seeking to terminate it.. The conventional approach to mapping arrhythmias has been to identify the earliest ventricular activation, which is assumed to be at, or close, to the ventricular tachycardia origin. This approach works well in focal ventricular tachycardia or at surgery where a large endocardial resection is ...
Wide QRS tachycardia may be due to ventricular tachycardia (VT), supraventricular tachycardia (SVT) with aberrant conduction, or atrioventricular reentrant tachycardia (AVRT) with an accessory pathway. Children with wide QRS complex tachycardia may p
Introduction: Postural tachycardia syndrome (POTS) is a disorder of orthostatic intolerance characterized by excessive tachycardia of unknown etiology. Our objective in this study was to evaluate the correlation between C-fiber involvement as shown by skin biopsy and adrenergic cardiac metaiodobenzylguanadine (MIBG) uptake in POTS patients. Methods: Skin biopsies of 84 patients with POTS were examined by Protein Gene Product 9.5 (PGP9.5) immunohistochemistry and were compared with MIBG myocardial scintigraphy imaging data. Results: Mean intraepidermal nerve fiber (IENF) density was in the lower normal age-adjusted range, 7.2 ± 2.9/mm (normal ≥7/mm), and was slightly below the normal range in 45% of POTS patients. MIBG uptake was reduced in 21% of patients. Low IENF density correlated with reduced cardiac MIBG uptake (r = 0.39, P = 0.001). Conclusions: A subset of neuropathic POTS patients may harbor mild small fiber neuropathy with abnormalities of unmyelinated nerve fibers in the skin ...
Atrial tachycardia is a type of heart rhythm problem in which the hearts electrical impulse comes from an ectopic pacemaker (that is, an abnormally located cardiac pacemaker) in the upper chambers (atria) of the heart, rather than from the sinoatrial node, the normal origin of the hearts electrical activity. Atrial tachycardias can exhibit very regular (consistent) heart rates ranging typically from 140 to 220 beats per minute.[1] As with any other form of tachycardia (rapid heart beat), the underlying mechanism can be either the rapid discharge of an abnormal focus, the presence of a ring of cardiac tissue that gives rise to a circle movement (reentry),[2] or a triggered rapid rhythm due to other pathological circumstances (as would be the case with some drug toxicities, such as digoxin toxicity). Atrial tachycardia is a risk factor for atrial fibrillation, as the rapid rhythm can trigger or degrade into the lack of a rhythm.[3] All atrial tachycardias are by definition supraventricular ...
Providing information and support about postural orthostatic tachycardia syndrome (PoTS) for sufferers, medical professionals, family, and friends - aiming to raise awareness.
Providing information and support about postural orthostatic tachycardia syndrome (PoTS) for sufferers, medical professionals, family, and friends - aiming to raise awareness.
The morphology of the tachycardia depends on its cause and the origin of the re-entry electrical circuit in the heart. In monomorphic ventricular tachycardia, the shape of each heart beat on the ECG looks the same because the impulse is either being generated from increased automaticity of a single point in either the left or the right ventricle, or due to a reentry circuit within the ventricle. The most common cause of monomorphic ventricular tachycardia is scarring of the heart muscle from a previous myocardial infarction (heart attack). This scar cannot conduct electrical activity, so there is a potential circuit around the scar that results in the tachycardia. This is similar to the re-entrant circuits that are the cause of atrial flutter and the re-entrant forms of supraventricular tachycardia. Other rarer congenital causes of monomorphic VT include right ventricular dysplasia, and right and left ventricular outflow tract VT. Polymorphic ventricular tachycardia, on the other hand, is most ...
Objectives: Patients with postural tachycardia syndrome (POTS) often appear anxious and report inattention. Patients with POTS were formally assessed for psychiatric disorders and inattention and compared with patients with attention deficit hyperactivity disorder (ADHD) and control subjects.. Methods: Patients with POTS (n = 21), ADHD (n = 18) and normal control subjects (n = 20) were assessed for DSM-IV psychiatric disorders and completed a battery of questionnaires that assessed depression, anxiety and ADHD characteristics.. Results: Patients with POTS did not have an increased prevalence of major depression or anxiety disorders, including panic disorder, compared with the general population. Patients with POTS had mild depression. They scored as moderately anxious on the Beck Anxiety Inventory but did not exhibit a high level of anxiety sensitivity. Patients with POTS scored significantly higher on inattention and ADHD subscales than control subjects. These symptoms were not present during ...
What is ventricular tachycardia? Ventricular tachycardia is an arrhythmia represented by rapid heartbeats that originate in the ventricles. During ventricular tachycardia the heart may beat inefficiently and as a result the blood pressure may decrease very much. This type of tachycardia may lead to life-threatening arrhythmias and cardiac arrest. Treatment options for ventricular tachycardias include medications, catheter ablation or placement of an implantable cardioverter-defibrillator (ICD).
Background: When noninvasive studies remain negative, the diagnosis of unexplained tachycardia in the youth is a dilemma. Invasive electrophysiological study (EPS) requires generally anesthesia and hospitalization in young children. The purpose of the study was to determine the factors of negativity of transesophageal EPS in children and teenagers complaining of tachycardia.. Population: 267 children, aged from 6 to 19 years, mean 15±3, complained of tachycardia and had a normal ECG in sinus rhythm.. Methods: Transesophageal EPS was performed in out-patient clinic without sedation but in presence of parents; EPS consisted of atrial pacing and atrial stimulation with 1 and 2 extrastimuli in control state and after isoproterenol. Long-term Holter monitoring was systematic in patients with negative EPS.. Results: Supraventricular tachycardia (SVT)(AV node re-entrant tachycardia, AV re-entrant tachycardia, atrial tachycardia) was induced in 146 patients (group I) and EPS remained negative in 121 ...
Ventricular tachycardia is the term for a fast heart rate that begins in the ventricles, the lower section of the heart. It leads to a fast yet regular heart rhythm. However, it can lead to ventricular fibrillation, which is a fast yet irregular heart beat. With this, the heart may beat so quickly and irregularly that it actually stops pumping blood. This can lead to sudden cardiac arrest and death. However, with treatment ventricular tachycardia can be handled and wont develop into ventricular fibrillation.. At times, especially when it develops in children and teens, it can be difficult if not impossible to identify the cause of ventricular tachycardia. However, in most cases, some sort of heart disease is the base cause. This may include a congenital heart defect, a previous heart attack, myocarditis, or dilated or hypertrophic cardiomyopathy. At times, ventricular tachycardia develops after a person has heart surgery. Some medications can also cause ventricular tachycardia. These include ...
Introduction. Neonatal tachycardia is defined as a resting heart rate (HR) of 182 beats per minute (bpm) when the baby is not crying.1,2 Supraventricular tachycardia (SVT) is the most common tachyarrhythmia in the neonatal period and usually presents with a HR of more than 200bpm and with a narrow QRS complex. In 80% of patients, it originates through a mechanism of retrograde conduction via an accessory pathway between the ventricle and the atrium, with an abrupt onset and termination. In 15% of patients it results from atrial ectopic beats, while 5% of cases correspond to re-entrant nodal tachycardias.3,4 Approximately half of these patients present with heart failure, with suspicion of sepsis in some cases. The tachycardia may be detected in the foetal period, most frequently between weeks 28 and 33 of gestation, possibly manifesting with hydrops.5. The key element in the management of haemodynamically unstable patients with tachycardia is electric cardioversion. However, in stable patients, ...
TY - JOUR. T1 - Use of the impella™ microaxial blood pump for ablation of hemodynamically unstable ventricular tachycardia. AU - Abuissa, Hussam. AU - Roshan, John. AU - Lim, Bernard. AU - Asirvatham, Ssamuel J.. PY - 2010/4. Y1 - 2010/4. N2 - Impella™ for VT Ablation. Ablation for ventricular tachycardia remains a challenge with suboptimal procedural success rates. One of the major causes of difficulty is precipitous hypotension when ventricular tachycardia is induced precluding even rapid mapping of the arrhythmia. We report the successful use of the Impella™ microcirculatory axial blood flow pump in 3 patients with hemodynamically unstable ventricular tachycardia that allowed successful completion of the procedure. In these 3 patients, there was no evidence of Impella™-related valvular disturbance, iatrogenic ventricular arrhythmias, or interference with mapping and ablation catheter movement. (J Cardiovasc Electrophysiol, Vol. 21, pp. 458-4, April 2010). AB - Impella™ for VT ...
TY - JOUR. T1 - Optimal surface electrocardiogram lead for identification of the mechanism of supraventricular tachycardia in children. AU - Liberman, Leonardo. AU - Pass, Robert H.. AU - Starc, Thomas J.. PY - 2008/1/1. Y1 - 2008/1/1. N2 - OBJECTIVE: Although supraventricular tachycardia (SVT) can be identified from any lead of the bedside monitor, the mechanism of tachycardia is not always obvious. We analyzed the 12-lead electrocardiogram (ECG) in SVT of pediatric patients with different mechanisms of SVT to determine if there is a consistent optimal lead for rhythm identification. METHODS: Twelve-lead ECGs during SVT were available for retrospective analysis in 54 patients. The tachycardia mechanism was determined either by intracardiac or transesophageal recording, or after cardioversion analysis of atrial flutter or fibrillation. Blinded analysis of each separate lead of the 12-lead ECG was done to determine the best lead to diagnose the mechanism of tachycardia. For statistical analysis, ...
A closed-heart method for treating ventricular tachycardia in a myocardial infarct patient afflicted with ventricular tachycardia is disclosed. The method comprises, first, defining a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) of the patient, and then ablating the thin layer of spared myocardial tissue by a closed-heart procedure with an ablation catheter. Apparatus for carrying out the method is also disclosed. Also disclosed is a method for prognosing the likelihood of ventricular tachycardia occuring in a myocardial infarct patient not previously diagnosed as afflicted with ventricular tachycardia. The method comprises detecting a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) in the patient.
A closed-heart method for treating ventricular tachycardia in a myocardial infarct patient afflicted with ventricular tachycardia is disclosed. The method comprises, first, defining a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) of the patient, and then ablating the thin layer of spared myocardial tissue by a closed-heart procedure with an ablation catheter. Apparatus for carrying out the method is also disclosed, Also disclosed is a method for prognosing the likelihood of ventricular tachycardia occuring in a myocardial infarct patient not previously diagnosed as afflicted with ventricular tachycardia. The method comprises detecting a thin layer of spared myocardial tissue positioned between the myocardial infarct scar tissue and the inner surface of the myocardium (the endocardium) in the patient.
STARS PO Box 175 Stratford-upon-Avon Warwickshire CV37 8YD +44 (0) 1789 450 564 [email protected] www.stars.org.uk Postural Tachycardia Syndrome (PoTS) This information sheet is designed for patients who have been diagnosed with postural tachycardia syndrome (PoTS). It contains information on the diagnosis, treatment and management of PoTS. Postural tachycardia syndrome (PoTS) is an abnormality of functioning of the autonomic (involuntary) nervous system. It is defined as an increase in heart rate of over 30 beats per minute (or to higher than 120 beats per minute) (40 bpm in those age 12-19) when standing upright. Typically there is no postural fall in blood pressure, although fainting (syncope) can occur (see below). The symptoms can vary (see below) but mostly involve orthostatic intolerance (the inability to withstand the upright posture) and are often made worse by various daily activities, e.g., modest physical exertion or food. It is a disorder which is slowly being recognised by the ...
Re-entrant atrial tachycardia is usually seen after cardiac surgery or catheter ablation with linear lesions that result in islets of scars. In patients with structurally normal hearts, atrial tachycardia is associated with a low mortality rate. Objectives: Ann Intern Med. My cardiologist now tells me this is common in about 50% of patients and is most likely a temporary issue. It is usually a self-limiting condition, but can cause serious haemodynamic deterioration in the immediate postoperative phase .JET is a narrow QRS complex tachycardia, usually with atrioventricular dissociation and slower atrial than ventricular rate. JAMA. I recently (August 2018) had my 1st ablation at TCA in Austin TX. 2017;92(1):98-105. Arrhythmias are common after cardiac surgery such as coronary artery bypass grafting surgery. Dual-loop circuits in postoperative atrial macro re-entrant tachycardias. 2007;89:91-5. reported that the most common site of focal ATs after AF ablation was the previously isolated PV. ...
Ventricular tachycardia is a very rare fetal arrhythmia accounting for fewer than 2% of fetal tachycardias. We describe a fetus presenting at 30 weeks gestation with ventricular tachycardia at a rate of 220 beats per min and fetal hydrops. The tachycardia was unresponsive to flecainide but was co...
Looking for Sinus tachycardia? Find out information about Sinus tachycardia. see arrhythmia arrhythmia , disturbance in the rate or rhythm of the heartbeat. Various arrhythmias can be symptoms of serious heart disorders; however,... Explanation of Sinus tachycardia
Looking for online definition of auricular tachycardia in the Medical Dictionary? auricular tachycardia explanation free. What is auricular tachycardia? Meaning of auricular tachycardia medical term. What does auricular tachycardia mean?
The heart has its own electrical system that controls the heart rate and contraction of the heart, which pumps blood throughout the body. Normal heart rates vary based on a persons age.. A rapid heart rate can be caused by many different factors. Sometimes tachycardia is the bodys response to a trigger like fever, anxiety, exercise, or pain, just to name a few. It also can be due to an abnormal rhythm in the heart (called a dysrhythmia), such as supraventricular tachycardia (SVT).. Congenital heart abnormalities, high blood pressure, medications, dehydration, caffeine, smoking, and recreational drug use also can cause tachycardia. Tachycardias can lead to dizziness, shortness of breath, chest pain, fainting, and heart palpitations (a sensation of feeling the heart beat fast or irregularly). Some tachycardias are life threatening and can lead to blood clots, causing a stroke, heart failure, and sudden death.. ...
The subjects are patients with COI and adult healthy volunteers of similar age, gender, and body mass.. Participation in this protocol is offered to people 18 years old or older, independently of gender, race, age, ethnicity, religion, or any other demographic or sociopolitical classifications.. The subjects in the second off-site study are family members of the identified proband. Participation in this protocol is offered to people 18 years old or older, independently of gender, race, ethnicity, religion, or any other demographic or sociopolitical classifications.. In the second off-site study, participation is also offered to subjects 10 to 17 years old, provided that appropriate informed consent/assent has been given by the subject and his/her parent or guardian.. EXCLUSION CRITERIA:. Age: Minors younger than 18 years old are excluded, except for the off-site study. Advanced age does not constitute an exclusion criterion. For subjects more than 55 years old, carotid Doppler studies are done, ...
This is an irregular narrow complex rhythm. The first 2 sinus P waves (S) are of the same morphology with a PRI of about 0.20 sec and a rate of about 60 bpm (lead II). The tachycardia starts with a premature complex with different P wave morphology (blue arrow). The tachycardia is best observed in V1. The rate tends to increase (107 to about 170 bpm) then there is a non-conducted P wave (first red arrow). The first conducted P has a shorter PRI compared to the previous PRI. The tachycardia resumes with 1:1 conduction then a non-conducted P wave is seen (second red arrow). Thus, this is atrial tachycardia with a block (type I block). ...
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Researchers discover autoantibodies in POTS patients that may account for increased circulation of norepinephrine and tachycardia upon standing.
TY - JOUR. T1 - Mode of induction of ventricular tachycardia and prognosis in patients with coronary disease. T2 - The multicenter unsustained tachycardia trial (MUSTT). AU - Piccini, Jonathan P.. AU - Hafley, Gail E.. AU - Lee, Kerry L.. AU - Fisher, John D.. AU - Josephson, Mark E.. AU - Prystowsky, Eric N.. AU - Buxton, Alfred E.. PY - 2009/8/1. Y1 - 2009/8/1. N2 - Mode of Induction in MUSTT. Introduction: Programmed stimulation is an important prognostic tool in the evaluation of patients with an ejection fraction ≤40% after myocardial infarction. Many believe that ventricular tachycardia (VT) requiring 3 ventricular extrastimuli (VES) for induction is less likely to occur spontaneously and has less predictive value. However, it is unknown whether the mode of VT induction is associated with long-term prognosis. Methods and Results: We analyzed a cohort of 371 patients enrolled in MUSTT who had inducible monomorphic VT and who were not treated with antiarrhythmic drugs or an implantable ...
POTS is a relaytively common cause of disabilty in young people, characterized by symptoms associated with fast heartbeat (tachycardia) upon standing.
With monomorphic ventricular tachycardia all of the QRS waves will likely be symmetrical. Every ventricular impulse is being created from the identical place in the ventricles thus all of the QRS
Case report A 31 year old female was admitted to A/E 6 hours after ingestion of 90 bupropion tablets (13.5g), with alcohol. Initially, the patient was conscious but disorientated, GCS 12/15, agitated, tachycardia (124 beats per minute), raised blood pressure (180/96), tremor, dystonia, increased muscle tone with brisk tendon reflexes and clonus. Activated charcoal was administered but the patient had a convulsion and aspirated. She deteriorated further with recurrent seizures, GCS 5/15, BP 120/60 and heart rate 120 beats per minute and required intubation, ventilation and sedation with diazepam. The ECG showed broad complex tachycardia, which reverted to normal sinus rhythm after treatment with adenosine 12mg iv (Figure 1). The patient was ventilated for 2 days during which she remained hypotensive and had extrapyramidal signs. She was extubated after 3 days. Discussion Bupropion, an atypical antidepressant is structurally similar to amphetamine and diethylpropion.2 It was originally approved ...
Dive into the research topics of Outer loop and isthmus in ventricular tachycardia circuits: Characteristics and implications. Together they form a unique fingerprint. ...
During wide complex tachycardia (heart rate , 100/min, QRS , 0.12 sec) the differentiation between supraventricular and ventricular origin of the arrhythmia is important to guide therapy. Several algorithms have been developed to aid in this differentiation (below). It is important to keep in mind that a good estimate of VT versus SVT can be made based on the clinical vignette: ...
Tachycardia induced cardiomyopathy is a potentially lethal cause of heart failure generally because of atrial tachycardia and less frequently ventricular tachycardia. We present two cases of Marines with severe traumatic blast injuries secondary to i
There are 2 main new findings in the present study. The first one is that, after removing autonomic influences, inhibition of endogenous NO synthesis with L-NMMA results in significant bradycardia. This effect is not explained by its pressor effect or baroreflex responses, as the same increase in SBP with phenylephrine did not reduce HR during autonomic blockade. These data show that NO has a tonic effect on human HR in vivo, and that this effect is independent of the autonomic control of HR. It is well known that NO is produced within the heart not only by endothelial cells, but also by myocytes.20,21 It has been proposed that NO has a role in the regulation of cardiac function both under normal and pathological conditions.11,22,23 NOS inhibition was previously shown to reduce HR in patients with transplanted, and presumably denervated, hearts.24 Our findings indicate for the first time that endogenous NO tonically regulates HR in healthy subjects. This is also in agreement with studies in ...
Symptomatic falls in blood pressure after standing or eating are a frequent clinical problem, particularly in the elderly. The symptoms are often due to cerebral hypoperfusion and include generalized weakness, dizziness or lightheadedness, visual blu
The heartbeat is controlled by the electrical system of the heart. This system is made up of several parts that tell the muscle of the heart when to contract. The SA node starts the heartbeat, causing the atria, or upper chambers of the heart, to contract. The signal then travels through the AV node, bundle of His, bundle branches, and Purkinje fibers. This causes the ventricles, the lower chambers of the heart, to contract. This organized flow of electrical signals produces a normal heartbeat. Normal heartbeats can be seen in an Electrocardiogram or ECG.. Focal Ventricular Tachycardia is a heart arrhythmia, or irregular heartbeat. During focal ventricular tachycardia, accelerated abnormal electrical pulses in the lower chambers, or ventricles, disrupt the normal firing of the SA node, causing the heart to beat rapidly. The abnormal signals originate in a specific area in the ventricles called a focus. This irregular heartbeat can be seen on an electrocardiogram.. A rapid heartbeat does not give ...
Ventricular tachycardia (VT) is a type of regular, fast heart rate that arises from improper electrical activity in the ventricles of the heart.. ​. Ventricular tachycardia may last for only a few seconds, or it can last for much longer. You may feel dizzy or short of breath, or have chest pain.. Although a few seconds may not result in problems, longer periods are dangerous.. ​. Sometimes, ventricular tachycardia can cause your heart to stop (cardiac arrest), which is a life-threatening medical emergency.. ​. Many different conditions can cause ventricular tachycardia. Its important to get a prompt, accurate diagnosis and appropriate care.. ​. The best way to prevent ventricular tachycardia is to treat or eliminate risk factors that may lead to heart disease. If you already have heart disease, follow your treatment plan and a heart-healthy lifestyle.. See your doctor if you have any problems with the heartbeat. In some cases, urgent care is needed. ...
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Ventricular Tachycardia Treatment in Frisco - Ventricular Tachycardia is the issue related to the heart. If we talk about Texas, many hospitals provide the be
Tachycardia[edit]. Main article: Tachycardia. Tachycardia is a resting heart rate more than 100 beats per minute. This number ... "Tachycardia, Fast Heart Rate". Tachycardia. American Heart Association. 2 May 2013. Retrieved 21 May 2014.. ... The American Heart Association states the normal resting adult human heart rate is 60-100 bpm.[1] Tachycardia is a fast heart ... Initially, both hyponatremia (low sodium levels) and hypernatremia (high sodium levels) may lead to tachycardia. Severely high ...
... can be classified based on its morphology: *Monomorphic ventricular tachycardia means that the ... RVOT tachycardia is a type of monomorphic ventricular tachycardia originating in the right ventricular outflow tract. RVOT ... Various diagnostic criteria have been developed to determine whether a wide complex tachycardia is ventricular tachycardia or a ... It may be very difficult to differentiate between ventricular tachycardia and a wide-complex supraventricular tachycardia in ...
Also, neuromuscular blockers may facilitate histamine release, which causes hypotension, flushing, and tachycardia. ... leading to hypotension and tachycardia. This muscarinic blocking effect is related to the acetylcholine moiety on the A ring on ...
Tachycardia. *Changes in blood pressure. Interactions[edit]. Because of its reliance on CYP3A4, reboxetine O-desethylation is ...
tachycardia. *urinary retention. *cutaneous vasodilation. [3] Clinically the most significant feature is delirium, particularly ...
"Atrial tachycardia". ECG Interpretation: An Incredibly Easy! Pocket Guide (4th ed.). Lippincott Williams & Wilkins. 2007. pp. ... Acute occurrence is usually non-life-threatening, but chronic occurrence can progress into tachycardia,[1] bradycardia or ... Phibbs, B. (1963). "Paroxysmal Atrial Tachycardia with Block Around the Ectopic Pacemaker: Report of a Case". Circulation. 28 ( ...
Another possible complication is "pacemaker-tracked tachycardia," where a supraventricular tachycardia such as atrial ... a b Pacemaker-Mediated Tachycardia at eMedicine *^ Transvenous Lead Extraction: Heart Rhythm Society Expert Consensus on ... This is known as fast-pacing, overdrive pacing, or anti-tachycardia pacing (ATP). ATP is only effective if the underlying ... Some ICD devices can distinguish between ventricular fibrillation and ventricular tachycardia (VT), and may try to pace the ...
Kastor JA (1990). "Multifocal Atrial Tachycardia". N Engl J Med. 322 (24): 1713-1717. doi:10.1056/NEJM199006143222405. PMID ... "Evidence supporting a new rate threshold for multifocal atrial tachycardia". Clin Cardiol. 28 (12): 3561-3563. doi:10.1002/clc. ... or multifocal atrial tachycardia if the rate is over 100.[6] This appears particularly commonly in exacerbations of chronic ...
TachycardiasEdit. In adults and children over 15, resting heart rate faster than 100 beats per minute is labelled tachycardia. ... Tachycardia may result in palpitation; however, tachycardia is not necessarily an arrhythmia. Increased heart rate is a normal ... Tachycardia that is not sinus tachycardia usually results from the addition of abnormal impulses to the normal cardiac cycle. ... Right ventricular outflow tract tachycardia is the most common type of ventricular tachycardia in otherwise healthy individuals ...
... and other forms of supraventricular tachycardia (e.g., AV nodal reentrant tachycardia).[135] Adults who survived congenital ... which may make it more difficult to separate from other supraventricular tachycardias or ventricular tachycardia.[64] ... During AF, if all of the impulses from the atria passed through the AV node, there would be severe ventricular tachycardia, ... Have been also identified new genes involved in tachycardia (CASQ2) or associated with an alteration in cardiomyocyte ...
Catecholaminergic polymorphic ventricular tachycardia. References[edit]. *^ Katz, Arnold M. (2005). Physiology of the Heart ( ... GeneReviews/NCBI/NIH/UW entry on Catecholaminergic Polymorphic Ventricular Tachycardia. *Calsequestrin at the US National ... A lack of or mutation in CSQ2 has been directly associated with catecholamine-induced polymorphic ventricular tachycardia (CPVT ...
Tachycardia (rapid heart rate). The initial cause of HAPE is a shortage of oxygen caused by the lower air pressure at high ...
Catecholaminergic polymorphic ventricular tachycardia. References[edit]. *^ a b c d e f g h "Brugada syndrome". Genetics Home ... and catecholaminergic polymorphic ventricular tachycardia". Circulation: Arrhythmia and Electrophysiology. 4 (6): 958-64. doi: ... "High prevalence of concealed Brugada syndrome in patients with atrioventricular nodal reentrant tachycardia". Heart Rhythm. 12 ... are also more likely to experience rapid heart rates due to less dangerous arrhythmias such as AV nodal re-entrant tachycardia[ ...
To treat postural tachycardia syndrome. *As an antidote to anticholinergic poisoning. *To reverse the effect of non- ...
Both genders showed significant tachycardia.[51] Likewise, taurine administration to diabetic rabbits resulted in 30% decrease ...
Indications for Class Ia agents are supraventricular tachycardia, ventricular tachycardia, symptomatic ventricular premature ... "Ventricular Tachycardia Medication: Antiarrhythmics, Class IC". Retrieved 4 October 2017.. *^ Wood JN, Boorman J (2005). " ... Class Ib agents are indicated for the treatment of ventricular tachycardia and symptomatic premature ventricular beats, and ... and to treat wide complex hemodynamically stable tachycardias. Oral procainamide is no longer being manufactured in the US, but ...
This study only counted heart rhythms which can respond to defibrillator shocks (tachycardia).[69] A major reason for the ... Ventricular fibrillation (quiver) / ventricular tachycardia (rapid beat). 38%. 19%. 2.0. 27,653. 2001-10 ... namely ventricular fibrillation or pulseless ventricular tachycardia, rather than asystole or pulseless electrical activity. ...
Tachycardia and hypertension are sometimes also present. Because of the spasms, patients may become increasingly fearful, ...
Rapid heart beat: (tachycardia): the heart starts beating much faster than normal.[26]. ...
Tachycardia and hypertension are sometimes also present.[12] ...
Text is available under the Creative Commons Attribution/Share-Alike License and the GFDL; additional terms may apply. See Terms of Use for details ...
Another cause of orthostatic headaches is postural orthostatic tachycardia syndrome (POTS), a form of dysautonomia, which is ... Mokri, B; Low, P. A. (2003). "Orthostatic headaches without CSF leak in postural tachycardia syndrome". Neurology. 61 (7): 980- ... It is also occasionally the most prominent symptom of postural orthostatic tachycardia syndrome (POTS). Distinguishing POTS ... from a cerebrospinal fluid leak can be difficult, because the defining symptom of POTS, positional tachycardia, also occurs in ...
... postural orthostatic tachycardia syndrome, and autonomic neuropathy.[citation needed] ... "Psychiatric profile and attention deficits in postural tachycardia syndrome". Journal of Neurology, Neurosurgery & Psychiatry ...
The ECG may show a tachycardia with a prolonged QT interval, which has been noted in proton pump inhibitor-associated ... This decrease in intracellular potassium results in a tachycardia. Magnesium has an indirect antithrombotic effect upon ...
"Arrhythmogenic Munchausen syndrome culminating in caffeine-induced ventricular tachycardia". J Electrocardiol. 44 (2): 229-31. ...
Fast heartbeat (tachycardia). *Fast breathing (tachypnea). *Fever. *In the most serious cases, delirium or unconsciousness ...
The therapeutic level is 10-20 micrograms/mL blood; signs of toxicity include tremor, nausea, nervousness, and tachycardia/ ...
Another possible complication is "pacemaker-tracked tachycardia," where a supraventricular tachycardia is tracked by the ... This is known as fast-pacing, overdrive pacing, or anti-tachycardia pacing (ATP). ATP is only effective if the underlying ... a b eMedicine , Pacemaker-Mediated Tachycardia Author: Brian Olshansky, MD. Coauthor(s): Chirag M Sandesara, MD; Noel G Boyle, ... Some ICD devices can distinguish between ventricular fibrillation and ventricular tachycardia (VT), and may try to pace the ...
Tachycardia, sweating, intercostal retractions, and sternoclavicular retractions may occur as well. Patients who have an ...
In gross overdosage also associated with dyspnoea, tachycardia, disorientation and convulsions.[3] ...
... tachycardia Atrial tachycardia Multifocal atrial tachycardia Junctional tachycardia Wide complex Ventricular tachycardia, any ... Tachycardia >169 bpm 1-2 years: Tachycardia >151 bpm 3-4 years: Tachycardia >137 bpm 5-7 years: Tachycardia >133 bpm 8-11 years ... Tachycardia > 159 beats per minute (bpm) 3-6 days: Tachycardia >166 bpm 1-3 weeks: Tachycardia >182 bpm 1-2 months: Tachycardia ... AV nodal reentrant tachycardia (AVNRT) is the most common reentrant tachycardia. It is a regular narrow complex tachycardia ...
Paroxysmal tachycardia, Junctional ectopic tachycardia, Sinus tachycardia, Atrial tachycardia, AV nodal reentrant tachycardia. ... AV nodal reentrant tachycardia[edit]. AV nodal reentrant tachycardia (AVNRT) is the most common reentrant tachycardia. It is a ... Junctional tachycardia[edit]. Junctional tachycardia is an automatic tachycardia originating in the AV junction. It tends to be ... Ventricular tachycardia, any tachycardia that originates in the ventricles. *Any narrow complex tachycardia combined with a ...
TACHYCARDIA. Br Med J 1937; 1 doi: https://doi.org/10.1136/bmj.1.3966.74-a (Published 09 January 1937) Cite this as: Br Med J ...
... or tachycardia, can increase the risks of stroke, sudden cardiac arrest, and death. Drugs and medications can cause tachycardia ... Tachycardia refers to a fast resting heart rate, usually over 100 beats per minute. Tachycardia can be dangerous, depending on ... Some people with tachycardia may have no symptoms or complications. However, tachycardia significantly increases the risk of ... Other conditions that explain the tachycardia might also be detected. Complications. The complications of tachycardia include: ...
... as serious as a dangerous ventricular tachycardia. Under any circumstance where cardiac injury has occurred, a ventricular ... Other articles where Ventricular tachycardia is discussed: cardiovascular disease: Ventricular arrhythmia: … ... as serious as a dangerous ventricular tachycardia. Under any circumstance where cardiac injury has occurred, a ventricular ...
Catecholaminergic polymorphic ventricular tachycardia (CPVT) is an inherited arrhythmogenic disorder that causes syncopal ... Catecholaminergic polymorphic ventricular tachycardia (CPVT) is a rare but highly malignant inherited arrhythmia disorder. It ... Syncope or sudden death in CPVT is a consequence of more rapid or sustained ventricular tachycardia or ventricular fibrillation ... Catecholaminergic polymorphic ventricular tachycardia (CPVT) is an inherited arrhythmogenic disorder that causes syncopal ...
... or tachycardia, can increase the risks of stroke, sudden cardiac arrest, and death. Drugs and medications can cause tachycardia ... What is tachycardia?. The heart consists of two ventricles and two atria. Tachycardia occurs when these beat too fast. ... Tachycardia refers to a fast resting heart rate, usually over 100 beats per minute. Tachycardia can be dangerous, depending on ... Some people with tachycardia may have no symptoms or complications. However, tachycardia significantly increases the risk of ...
Ventricular tachycardia (VT) is a rapid heartbeat that starts in the lower chambers of the heart (ventricles). ... Ventricular tachycardia may not cause symptoms in some people. However, it can be deadly. It is a major cause of sudden cardiac ... Ventricular tachycardia (VT) is a rapid heartbeat that starts in the lower chambers of the heart (ventricles). ...
Propranolol decreases tachycardia and improves symptoms in the postural tachycardia syndrome: less is more. Circulation 2009; ... Acetylcholinesterase inhibition improves tachycardia in postural tachycardia syndrome. Circulation 2005; 111:2734. ... Postural tachycardia syndrome (POTS). Neurology 1995; 45:S19.. *Freeman R, Wieling W, Axelrod FB, et al. Consensus statement on ... Postural tachycardia syndrome (POTS). Circulation 2013; 127:2336.. *Ojha A, Chelimsky TC, Chelimsky G. Comorbidities in ...
Sinus Tachycardia. After a visit to your health care professional, you heard about sinus tachycardia and now, you want to know ... Tachycardia in Children. Tachycardia is when the heart beats faster than its normal pace. Learn about the effects it has on ... Tachycardia after Eating. Tachycardia or increased heart rate after consumption of regular food or food high in sugar or fat ... Tachycardia Causes. Tachycardia is an abnormally high heart rate. There are three types of this heart condition, which help to ...
Supraventricular tachycardia is a type of abnormal heart rhythm in which the heart beats very quickly. ... What Is Supraventricular Tachycardia (SVT)?. Supraventricular tachycardia (SVT) is a type of arrhythmia (abnormal heart rhythm ... Who Gets Supraventricular Tachycardia (SVT)?. Supraventricular tachycardia usually affects infants, young kids, and teens. ... Supraventricular tachycardia (soo-pruh-ven-TRIK-yuh-ler tak-ih-KAR-dee-uh) often happens suddenly and can last for a few ...
Catecholaminergic polymorphic ventricular tachycardia (CPVT) is a condition characterized by an abnormal heart rhythm ( ... it can trigger an abnormally fast heartbeat called ventricular tachycardia. Episodes of ventricular tachycardia can cause light ... Catecholaminergic Polymorphic Ventricular Tachycardia. Circ J. 2016 May 25;80(6):1285-91. doi: 10.1253/circj.CJ-16-0326. Epub ... Catecholaminergic polymorphic ventricular tachycardia (CPVT) is a condition characterized by an abnormal heart. rhythm ( ...
no matter which type of tachycardia you have, you may feel: *dizziness *lightheadedness *shortness of breath *chest pain *heart ...
... is another type of "short-circuit" arrhythmia. It may result either from atrio- ... ventricular nodal re-entrant tachycardia (AVNRT) or from an accessory pathway, which may occur as part of the Wolff-Parkinson- ... atrio-ventricular reciprocating tachycardia). Under rare circumstances, patients with Wolff-Parkinson-White syndrome can ...
... one will confuse the adrenaline caused high HR that stops in a few minutes with tachycardia. More and more I believe, aft... ... i have tachycardia. i am a 24 yr old female. i had svt since i was 17 had an ablation for it when i was 19. still have this ... i have tachycardia. i am a 24 yr old female. i had svt since i was 17 had an ablation for it when i was 19. still have this ... but the tachycardia causes my stress and definetly my anxiety also. i feel my heartbeat all th time. i feel it pound too. its ...
Tachycardia is a rapid heart rate caused by a problem in the hearts electrical system. Find out more about this common heart ... Sinus tachycardia. http://www.uptodate.com/home. Accessed Aug. 19, 2016.. *Marchlinski F. In: Harrisons Online. 18th ed. New ... Tachycardia - Fast heart rate. American Heart Association. http://www.heart.org/HEARTORG/Conditions/Arrhythmia/AboutArrhythmia/ ... Tachycardia-Fast-Heart-Rate_UCM_302018_Article.jsp#.V7s5Rmf2bIU. Accessed Aug. 22, 2016. ...
People with paroxysmal supraventricular tachycardia experience a faster-than-normal heart rate. Learn more about the symptoms, ... What are the risk factors for paroxysmal supraventricular tachycardia?. PSVT affects about 1 in every 2,500 children. It is the ... How is paroxysmal supraventricular tachycardia treated?. You might not need treatment if your symptoms are minimal or if you ... What are the symptoms of paroxysmal supraventricular tachycardia?. The symptoms of PSVT resemble the symptoms of an anxiety ...
Find Paroxysmal supraventricular tachycardia information, treatments for Paroxysmal supraventricular tachycardia and Paroxysmal ... MedHelps Paroxysmal supraventricular tachycardia Center for Information, Symptoms, Resources, Treatments and Tools for ... Posts on Paroxysmal supraventricular tachycardia. Paroxysmal afib and SVT like symptoms as well as Bradycardia - Heart Disease ... I have been diagnosed with Supraventricular Tachycardia along with a sinus arrhythmia. This... ...
The Postural Orthostatic Tachycardia Syndrome... by Dr. Jeffrey Boris - Duration: 1:03:58. UMNPediatrics 17,333 views ... Postural Tachycardia Syndrome (POTS) - Mayo Clinic - Duration: 5:21. Mayo Clinic 159,794 views ... Postural Orthostatic Tachycardia Syndrome (POTS) - Duration: 46:32. Dr. John Bergman 43,152 views ... Living with severe P.O.T.S. (postural orthostatic tachycardia syndrome) - Duration: 12:38. Molly Nason 21,668 views ...
Children with supraventricular tachycardia may have episodes that come and go suddenly. ... Supraventricular tachycardia is one type of fast heartbeat. ... Supraventricular tachycardia (SVT) is one type of tachycardia. ... What is supraventricular tachycardia? Supraventricular tachycardia (pronounced sue-prah-ven-TRIK-yu-lar tack-ih-CAR-dee-ah) is ... There are many different types of tachycardia. Sinus tachycardia is a normal increase in heart rate in response to activity ( ...
This group of tachycardias is the most common of the paroxysmal atrial mechanisms and is seen most frequently in women. In ... Ablation of the tachycardia was performed at the right angle intersection of lines drawn from the His and coronary sinus sites ... Ablation of the tachycardia was performed at the right angle intersection of lines drawn from the His and coronary sinus sites ... An example of AVNRT tachycardia is shown in Figure 1. The P waves are hidden within the QRS complex, as this is a slow-fast ...
Definition of fetal tachycardia. Provided by Stedmans medical dictionary and Drugs.com. Includes medical terms and definitions ...
What happens when ventricular tachycardia throws off the steady, coordinated rhythm of your heartbeat?. When the heart works ... In ventricular tachycardia, a misfire of your hearts electrical system throws the rhythm off. ... In certain settings, ventricular tachycardia may be associated with sudden death: most often in abnormal hearts - such as those ... Implants: For patients who are at risk for certain forms of ventricular tachycardia, an implantable cardioverter defibrillator ...
Paroxysmal atrial tachycardia is a type of irregular heartbeat that increases heart rate. Read on to learn about the causes, ... Paroxysmal atrial tachycardia (PAT) is also known as paroxysmal supraventricular tachycardia (PSVT). Other types of tachycardia ... What is paroxysmal atrial tachycardia?. Paroxysmal atrial tachycardia is a type of arrhythmia, or irregular heartbeat. ... Paroxysmal Atrial Tachycardia (PAT). Medically reviewed by Elaine K. Luo, MD - Written by Corinna Underwood - Updated on August ...
Atrial tachycardias have traditionally been characterized as automatic, triggered, or reentrant. However, the European Society ... Sinoatrial nodal reentrant tachycardia (SANRT), also called sinus node reentry or sinus node reentrant tachycardia, falls into ... Focal atrial tachycardia due to an automatic, triggered, or microreentrant mechanism. ●Macroreentrant atrial tachycardia, ... See Focal atrial tachycardia and Intraatrial reentrant tachycardia and Overview of atrial flutter.) ...
Supraventricular tachycardia is a series of rapid heartbeats that begin in or involve the upper chambers (atria) of the heart. ... Atrial Flutter & Atrial Tachycardia. Typical atrial flutter results from a single "short-circuit" in the right atrium. This ... Atrial Flutter & Atrial Tachycardia. An arrhythmia is an abnormality in the timing or pattern of the heartbeat. Arrhythmias may ... Some patients have atrial tachycardia, a rapidly firing focus which may originate from either atria. These arrhythmias also ...
A disruption in the normal electrical impulses controlling the pumping action of the heart causes tachycardia, according to ... What are some causes of supraventricular tachycardia?. A: Some causes of supraventricular tachycardia are problems with the ... Certain diseases can contribute to tachycardia, including hyperthyroidism and anemia, and tachycardia can be an accompanying ... A: A heart rate of more than 100 beats per minute in adults, a condition called tachycardia, is considered fast according to ...
The term tachycardia refers to a rapid heartbeat of over 100 beats per minute. Supraventricular tachycardia is frequently ... How Is Supraventricular Tachycardia Classified?. An SVT is classified medically on the basis of the path that the electrical ... How Is Supraventricular Tachycardia Treated?. Medications may be used to treat many patients with SVT. The most commonly used ... How Is Supraventricular Tachycardia Diagnosed?. The electrocardiogram (ECG or EKG) provides a picture of the heart rhythm and ...
Ventricular tachycardia can be classified based on its morphology: *Monomorphic ventricular tachycardia means that the ... RVOT tachycardia is a type of monomorphic ventricular tachycardia originating in the right ventricular outflow tract. RVOT ... Various diagnostic criteria have been developed to determine whether a wide complex tachycardia is ventricular tachycardia or a ... It may be very difficult to differentiate between ventricular tachycardia and a wide-complex supraventricular tachycardia in ...
In 1995, Kipel and colleagues reported a case of malignant wide complex tachycardia in a 10 year old boy who received adenosine ... Adenosine for the Management of Neonatal and Pediatric Supraventricular Tachycardia. Contributing Editor: Marcia L. Buck, Pharm ...
  • Paroxysmal supraventricular tachycardia (PSVT) is another type of "short-circuit" arrhythmia. (hopkinsmedicine.org)
  • What is paroxysmal supraventricular tachycardia? (healthline.com)
  • Episodes of faster-than-normal heart rate characterize paroxysmal supraventricular tachycardia (PSVT). (healthline.com)
  • What are the risk factors for paroxysmal supraventricular tachycardia? (healthline.com)
  • How is paroxysmal supraventricular tachycardia diagnosed? (healthline.com)
  • How is paroxysmal supraventricular tachycardia treated? (healthline.com)
  • This group of tachycardias is the most common of the paroxysmal atrial mechanisms and is seen most frequently in women. (medscape.com)
  • Paroxysmal atrial tachycardia is a type of arrhythmia, or irregular heartbeat. (healthline.com)
  • Paroxysmal atrial tachycardia (PAT) is also known as paroxysmal supraventricular tachycardia (PSVT) . (healthline.com)
  • Supraventricular tachycardia is frequently abbreviated as SVT (formerly paroxysmal atrial tachy- cardia or PAT). (ahajournals.org)
  • Paroxysmal supraventricular tachycardia (PSVT) is episodes of rapid heart rate that start in a part of the heart above the ventricles. (softpanorama.org)
  • SVT is also called atrial tachycardia, paroxysmal supraventricular tachycardia (PSVT), or paroxysmal atrial tachycardia (PAT). (rexhealth.com)
  • Often, however, in a clinical setting, it is used loosely as a synonym for paroxysmal supraventricular tachycardia (PSVT), which refers to those SVTs that have a sudden, almost immediate onset. (bionity.com)
  • There is little known about the patient perspective in patients suffering from paroxysmal supraventricular tachycardia (PSVT), a condition with a sudden onset of fast heart rhythm. (his.se)
  • Paroxysmal supraventricular tachycardia (PSVT) is an abnormally fast heart beat, which begins and ends suddenly. (epnet.com)
  • Paroxysmal supraventricular tachycardia (PSVT) is more common in women. (epnet.com)
  • A diagnosis of paroxysmal supraventricular tachycardia (PSVT) usually begins when a person notices uncomfortable attacks of rapid heart beating. (epnet.com)
  • Treatment for paroxysmal supraventricular tachycardia (PSVT) involves stopping the electrical impulses causing the attacks. (epnet.com)
  • This type of rhythm includes paroxysmal atrial tachycardia (PAT). (doctorslounge.com)
  • Incidence of paroxysmal supraventricular tachycardia is approximately 1-3 per 1000. (doctorslounge.com)
  • Paroxysmal atrial tachycardia (PAT), especially with second-degree AV block (PAT with block) classically is associated with digitalis toxicity. (doctorslounge.com)
  • SVT's are considered as a general term for any tachycardias that originate above the atrioventricular node (AV node) while other physicians prefer to simply name each type of tachycardia and consider any SVT that occurs off and on as either a SVT or a paroxysmal SVT ( PSVT ). (emedicinehealth.com)
  • Supraventricular tachycardia is also called paroxysmal supraventricular tachycardia and abbreviated either SVT or PSVT. (emedicinehealth.com)
  • When tachycardia occurs, it is usually referred to as paroxysmal supraventricular tachycardia (often abbreviated PSVT). (emedicinehealth.com)
  • Wolff-Parkinson-White syndrome) Pacemaker-tracked or pacemaker-mediated tachycardia Tachycardias may be classified as either narrow complex tachycardias (supraventricular tachycardias) or wide complex tachycardias. (wikipedia.org)
  • Tachycardias may be classified as either narrow complex tachycardias (supraventricular tachycardias) or wide complex tachycardias. (wikipedia.org)
  • Although not a frequent type of arrhythmia, JET is one of the most serious and difficult-to-treat supraventricular tachycardias. (medscape.com)
  • Most automatic tachycardias are supraventricular tachycardias (SVT). (wikipedia.org)
  • Most supraventricular tachycardias have a narrow QRS complex on EKG , but it is important to realise that supraventricular tachycardia with aberrant conduction (SVTAC) can produce a wide-complex tachycardia that may mimic ventricular tachycardia (VT). (bionity.com)
  • The other supraventricular tachycardias are infrequently or rarely diagnosed. (emedicinehealth.com)
  • There are two semantic problems in the literature with supraventricular tachycardias (SVTs). (emedicinehealth.com)
  • BACKGROUND: Radiofrequency ablation (RFA) has been shown to be very effective in the treatment of supraventricular tachycardias and has replaced surgical ablation. (biomedsearch.com)
  • citation needed] Some causes of tachycardia include: Adrenergic storm Alcohol Amphetamine Anaemia Antiarrhythmic agents Anxiety Atrial fibrillation Atrial flutter Atrial tachycardia AV nodal reentrant tachycardia Brugada syndrome Caffeine Cocaine Exercise Fear Fever Hypoglycemia Hypovolemia Hyperthyroidism Hyperventilation Infection Junctional tachycardia Methamphetamine Multifocal atrial tachycardia Nicotine Pacemaker mediated Pain Pheochromocytoma Sinus tachycardia Tricyclic antidepressants Wolff-Parkinson-White syndrome An electrocardiogram (ECG) is used to classify the type of tachycardia. (wikipedia.org)
  • The upper limit of normal rate for sinus tachycardia is thought to be 220 bpm minus age. (wikipedia.org)
  • Sinus tachycardia is a condition that affects the sinus node of the heart and results in the increase of the resting heart rate, taking it above 100 beats per minute. (buzzle.com)
  • Inappropriate Sinus Tachycardia is a condition that is not very well understood, and is commonly seen in women. (buzzle.com)
  • What are the causes of sinus tachycardia? (webmd.com)
  • Sinus tachycardia is a normal increase in heart rate in response to activity (running or playing), fear or excitement. (seattlechildrens.org)
  • Often sinus tachycardia is a normal response to certain situations such as exercise, anxiety, distress, or fever. (innerbody.com)
  • Certain disorders such as thyroid disease, anemia, and low blood pressure are also associated with sinus tachycardia. (innerbody.com)
  • I never do narcotics, I don't use any medicine -- still, I got sinus tachycardia, now my heart beats 90 bpm or more every single day for nearly a month, except when I sleep. (medhelp.org)
  • I went to the er last week and I have inappropriate sinus tachycardia. (healthboards.com)
  • The different types are the following:[citation needed] Sinus tachycardia may be considered an automatic tachycardia, since the sinoatrial node (SAN) is discharging at an abnormally fast rate. (wikipedia.org)
  • This definition includes sinus tachycardia . (bionity.com)
  • Sinus tachycardia is considered "appropriate" when a reasonable stimulus, such as the catecholamine surge associated with fright, stress, or physical activity, provokes the tachycardia. (bionity.com)
  • It is therefore impossible to distinguish on the EKG from ordinary sinus tachycardia. (bionity.com)
  • Atrial tachycardias have traditionally been characterized as automatic, triggered, or reentrant. (uptodate.com)
  • In 2015, the joint American College of Cardiology, American Heart Association, and Heart Rhythm Society guidelines further defined sinus node reentrant tachycardia as 'a specific type of focal atrial tachycardia that is due to microreentry arising from the sinus node complex, characterized by abrupt onset and termination, resulting in a P-wave morphology that is indistinguishable from sinus rhythm' [ 2 ]. (uptodate.com)
  • Sinoatrial nodal reentrant tachycardia (SANRT), also called sinus node reentry or sinus node reentrant tachycardia, falls into the latter group of macroreentrant arrhythmias. (uptodate.com)
  • See 'Focal atrial tachycardia' and 'Intraatrial reentrant tachycardia' and 'Overview of atrial flutter' . (uptodate.com)
  • See 'Intraatrial reentrant tachycardia' . (uptodate.com)
  • One type of SVT (AV nodal reentrant tachycardia or AVNRT) occurs because the electrical impulse travels in a circle using extra fibers in and around the AV node ( Figure 2 ). (ahajournals.org)
  • Horie K, Otomo K, Mori S, Kikuchi Y, Meguro T. Uncommon presentation of drug-refractory pacemaker-mediated common atrioventricular nodal reentrant tachycardia and a simple solution by reprogramming. (medscape.com)
  • We report a case of bundle branch reentrant ventricular tachycardia as primary manifestation of myotonic dystrophy and discuss associated diagnostic and treatment challenges. (hindawi.com)
  • Atrioventricular nodal reentrant tachycardia (AVNRT). (rexhealth.com)
  • These tachycardias, or fast heart rhythms, differ from reentrant tachycardias (AVRT and AVNRT) in which there is an abnormal electrical pathway which gives rise to the pathology. (wikipedia.org)
  • It is important to recognise an automatic tachycardia because the treatment will be different to that for a reentrant tachycardia. (wikipedia.org)
  • This means that whereas a reentrant tachycardia will both begin and end abruptly as cardiac conduction utilises then ceases to utilise the accessory pathway, an automatic tachycardia will rise and fall gradually in rate as the automatic focus increases and decreases its automatic rate of electrical discharge. (wikipedia.org)
  • PSVTs are usually AV nodal reentrant tachycardias . (bionity.com)
  • Sinoatrial node reentrant tachycardia (SANRT) is caused by a reentry circuit localised to the SA node, resulting in a normal-morphology p-wave that falls before a regular, narrow QRS complex. (bionity.com)
  • An electrocardiogram (ECG) is used to classify the type of tachycardia. (wikipedia.org)
  • Supraventricular tachycardia (SVT) is one type of tachycardia. (seattlechildrens.org)
  • Tachycardia can be ventricular (in the lower chambers of the heart) or atrial (in the upper chambers of the heart), and the treatment strategy may vary depending on what type of tachycardia one may have. (medtronic.com)
  • Ventricular tachyarrhythmia (ventricular fibrillation) is the most dangerous type of tachycardia. (biotronik.com)
  • A narrow complex tachycardia with an accessory conduction pathway, often termed "supraventricular tachycardia with pre-excitation" (e.g. (wikipedia.org)
  • Supraventricular tachycardia (SVT) is caused by abnormalities of impulse conduction (re-entrant tachycardias) or disorders of impulse initiation (automatic tachycardias), causing a narrow complex tachycardia. (doctorslounge.com)
  • It usually has narrow complex tachycardia but this is not always the. (bioportfolio.com)
  • The initial ECG demonstrated a narrow complex tachycardia (Figure 1 ). (hindawi.com)
  • ECG showing narrow complex tachycardia with a long RP pattern. (hindawi.com)
  • An extra pathway, often present in supraventricular tachycardia, can lead to the abnormally fast heartbeat of PSVT. (healthline.com)
  • Because sinus tachycardias (and some other SVTs) have a gradual (i.e. non-immediate) onset, they are excluded from the PSVT category. (bionity.com)
  • People with PSVT have attacks of tachycardia that can last anywhere from a few minutes to several hours. (epnet.com)
  • What is Supraventricular Tachycardia (SVT, PSVT Definitions)? (emedicinehealth.com)
  • It is usually a regular, wide complex tachycardia with a rate between 120 and 250 beats per minute. (wikipedia.org)
  • In 1995, Kipel and colleagues reported a case of malignant wide complex tachycardia in a 10 year old boy who received adenosine two days after he had undergone a Fontan procedure. (medscape.com)
  • In the clinical setting, it is important to determine whether a wide-complex tachycardia is an SVT or a ventricular tachycardia, since they are treated differently. (bionity.com)
  • A number of different algorithms have been devised to determine whether a wide complex tachycardia is supraventricular or ventricular in origin. (bionity.com)
  • This and the following steps are based on Brugada's algorithm for diagnosis of wide-complex tachycardia. (anaesthetist.com)
  • Ventricular tachycardia (VT or V-tach) is a potentially life-threatening cardiac arrhythmia that originates in the ventricles. (wikipedia.org)
  • Catecholaminergic polymorphic ventricular tachycardia (CPVT) is a rare but highly malignant inherited arrhythmia disorder. (springer.com)
  • The presence of this second connection between the atria and ventricles is a setup for developing a "short-circuit" arrhythmia: electrical impulses may start traveling in a circular pattern and cause the heart to beat too rapidly (AVRT: atrio-ventricular reciprocating tachycardia). (hopkinsmedicine.org)
  • http://www.heart.org/HEARTORG/Conditions/Arrhythmia/AboutArrhythmia/Tachycardia-Fast-Heart-Rate_UCM_302018_Article.jsp#.V7s5Rmf2bIU. (mayoclinic.org)
  • I have been diagnosed with Supraventricular Tachycardia along with a sinus arrhythmia. (medhelp.org)
  • In addition, nonparoxysmal junctional tachycardia is a related but rare pattern of arrhythmia that can be observed in the setting of digoxin toxicity. (medscape.com)
  • Tachycardia is a type of arrhythmia where the electrical signals controlling the heart travel across the heart faster than normal, triggering the heart to beat too rapidly. (innerbody.com)
  • Because of the typical pattern of arrhythmias (bidirectional ventricular tachycardia and the occurrence and severity of arrhythmia correlated well with exercise workload) during exercise stress test, CPVT can be identified promptly. (nih.gov)
  • An automatic tachycardia is a cardiac arrhythmia which involves an area of the heart generating an abnormally fast rhythm, sometimes also called enhanced automaticity. (wikipedia.org)
  • Due to tachycardia, heart generates lethal arrhythmia beats namely atrial flutter (AFL), atrial fibrillation (A-Fib), and ventricular fibrillation (V-Fib). (springer.com)
  • Focal atrial tachycardia is a relatively uncommon arrhythmia. (bmj.com)
  • Arrhythmogenic Right Ventricular Cardiomyopathy (ARVC) is an inherited arrhythmia disorder with high risk of ventricular tachycardia or fibrillation, and implantable cardioverter defibrill. (bioportfolio.com)
  • Arrhythmia initiation in catecholaminergic polymorphic ventricular tachycardia type 1 depends on both heart rate and sympathetic stimulation. (bioportfolio.com)
  • Catecholaminergic polymorphic ventricular tachycardia (CPVT) is often a life-threatening arrhythmia disorder with variable penetrance and expressivity. (bioportfolio.com)
  • It may result either from atrio-ventricular nodal re-entrant tachycardia (AVNRT) or from an accessory pathway, which may occur as part of the Wolff-Parkinson-White (WPW) syndrome. (hopkinsmedicine.org)
  • [ 6 , 7 ] The AVNRT tachycardias are described as typical when the posterior input from the crista terminalis functions as the slow (slow-fast) pathway and the anterior or interatrial septum acts as the fast pathway. (medscape.com)
  • An example of AVNRT tachycardia is shown in Figure 1. (medscape.com)
  • SVT is most often due to the junctional tachycardias, atrioventricular nodal re-entrant tachycardia (AVNRT) or atrioventricular re-entrant tachycardia (AVRT). (doctorslounge.com)
  • In this study, an automated diagnosis support system (DSS) is developed for accurate discrimination and classification of complete classes of tachycardia beats (atrial as well as ventricular) using higher-order spectra (HOS). (springer.com)
  • Diagnosis of multiclass tachycardia beats using recurrence quantification analysis and ensemble classifiers. (springer.com)
  • Radiofrequency ablation of focal atrial tachycardia is extremely successful and this approach is becoming the preferred treatment for symptomatic patients.In this review, we describe the pathophysiology, anatomical localisation, clinical features, diagnosis and therapeutic options for the management of focal atrial tachycardia. (bmj.com)
  • A supraventricular tachycardia diagnosis is confirmed with an ECG. (emedicinehealth.com)
  • Guidelines for the diagnosis and management of Catecholaminergic Polymorphic Ventricular Tachycardia. (genome.jp)
  • Pacer arrhythmias: myopotential triggering of pacemaker mediated tachycardia. (medscape.com)
  • With the exception of beta-blockers, no pharmacologic therapy of proven effectiveness is available: although beta-blockers reduce the occurrence of ventricular tachycardia, 30% of patients treated with beta-blockers still experience cardiac arrhythmias and eventually require implantable cardioverter defibrillator implantation to prevent cardiac arrest. (nih.gov)
  • People with supraventricular tachycardia (SVT) often have an extra electrical circuit in their hearts and may occasionally experience very fast heart beats (arrhythmias) that are unrelated to exercise, fever or stress. (bidmc.org)
  • Moreover, focal atrial tachycardia can trigger other atrial arrhythmias like atrial fibrillation and flutter. (bmj.com)
  • In the course of understanding the mechanism and therapy of atrial tachycardia, important distinctions are emerging in electrophysiological evaluations of various atrial arrhythmias, which come under scrutiny in this latest addition to the CATA Series. (wiley.com)
  • Junctional ectopic tachycardia (JET) is characterized by rapid heart rate for a person's age that is driven by a focus with abnormal automaticity within or immediately adjacent to the atrioventricular (AV) junction of the cardiac conduction system (ie, AV node-His bundle complex). (medscape.com)
  • JET primarily occurs in 2 forms: idiopathic chronic junctional ectopic tachycardia, which is observed in the setting of a structurally normal heart, and transient postoperative junctional ectopic tachycardia occurs following repair of congenital heart disease. (medscape.com)
  • Spontaneous resolution of congenital junctional ectopic tachycardia (JET) has been observed in as many as one third of patients who reach age 1 year. (medscape.com)
  • Signs of ectopic pregnancy include: more common signs: pelvic tenderness adnexal tenderness abdominal tenderness other reported signs: cervical motion tenderness rebound tenderness or peritoneal signs pallor abdominal distension enlarged uterus tachycardia (more than 100 beats per minute) or hypotension (less than 100/ 60 mmHg) shock or collapse orthostatic hypotension. (tripdatabase.com)
  • Atrial ectopic tachycardia, in which the focus or foci are in the atria of the heart, is an automatic tachycardia. (wikipedia.org)
  • Junctional ectopic tachycardia, in which the focus is in the atrioventricular node (AVN), and Accelerated idioventricular rhythm, involving a ventricular focus, are also examples. (wikipedia.org)
  • I was diagnosed with ectopic Atrial Tachycardia with a possible innapropriate sinus rhythm. (dailystrength.org)
  • Unifocal) Atrial tachycardia is tachycardia resultant from one ectopic foci within the atria, distinguished by a consistent p-wave of abnormal morphology that fall before a narrow, regular QRS complex. (bionity.com)
  • Atrial tachycardia typically arises from an ectopic source in the atrial muscle and produces an atrial rate of 150-250 beats/min, slower than that of atrial flutter. (doctorslounge.com)
  • The left atrial appendage (LAA) is an uncommon site for origin of ectopic atrial tachycardia [ 1 ]. (hindawi.com)
  • We present an unusual case of an otherwise healthy 7-year-old girl who presented with an ectopic atrial tachycardia arising from the left atrial appendage and underwent radiofrequency ablation on ECMO support. (hindawi.com)
  • In general, a resting heart rate over 100 beats per minute is accepted as tachycardia in adults. (wikipedia.org)
  • Tachycardia refers to a fast resting heart rate, usually over 100 beats per minute. (medicalnewstoday.com)
  • Tachycardia is an abnormal heart rate condition, where the rate of the heartbeats go overboard, i.e. over a count of 100 beats in a minute. (buzzle.com)
  • Heart disease can damage the tissues of the heart and cause tachycardia, which is a condition characterized by a rested heart rate higher than the normal 60 to 100 beats per minute. (reference.com)
  • A heart rate of more than 100 beats per minute in adults, a condition called tachycardia, is considered fast according to The American Heart Association. (reference.com)
  • The term tachycardia refers to a rapid heartbeat of over 100 beats per minute. (ahajournals.org)
  • A heart rate greater than 100 beats per minute is termed tachycardia. (innerbody.com)
  • In newborns and infants, tachycardia is defined as a heart rate greater than 150 beats per minute. (innerbody.com)
  • Tachycardia is a fast heart rate - more than 100 beats per minute - that can either start in the heart's lower chambers (ventricles) or upper chambers (atria). (medtronic.com)
  • Tachycardia occurs when your heart rate rises above 100 beats per minute. (livestrong.com)
  • Supraventricular tachycardia (SVT) means that from time to time your heart beats very fast for a reason other than exercise, high fever, or stress. (rexhealth.com)
  • The normal heart rate is considered to be 60 to 100 beats/min, with tachycardia the rate exceeds 100 beats/min. (patientslikeme.com)
  • Ventricular tachycardia is a condition, wherein the pulse rate is more than 100 beats per minute with minimum three continuous irregular heartbeats. (openpr.com)
  • But if you have supraventricular tachycardia, or SVT, it means your heart beats faster than normal-usually more than 100 beats per minute. (cardiosmart.org)
  • The term tachycardia refers to a rapid heart rate , routinely understood as greater than 100 beats per minute. (wisegeek.com)
  • It is thought by experts that a sustained event of pulseless ventricular tachycardia has often been preceded by short, self-limited runs - six beats or less - on one or more occasions. (wisegeek.com)
  • Automatic atrial tachycardia is typically associated with heart rates ranging from 150-200 beats/minute. (doctorslounge.com)
  • In most patients with ventricular tachycardia the rate is in the range of 170 beats per minute or more. (rochester.edu)
  • Supraventricular tachycardia is a rapid heart rate (tachycardia, or a heart rate above 100 beats per minute) that is caused by electrical impulses that originate above the heart's ventricles. (emedicinehealth.com)
  • Tachycardia is a very fast heart rate - over 100 beats per minute. (biotronik.com)
  • In ventricular tachycardia, the heart beats rhythmically and very quickly. (biotronik.com)
  • Tachycardia is defined as a heart rate greater than 100 beats per minute, while a normal heart beats is measured at 72 beats per minute. (consumerinjurylawyers.com)
  • Patients with Tachycardia may often feel out of breath because the faster the heart beats, the more oxygen it requires. (consumerinjurylawyers.com)
  • Tachycardia refers to any heartbeat greater than 100 beats per minute. (biotronik.com)
  • Atrioventricular reciprocating tachycardia (AVRT), including Wolff-Parkinson-White syndrome. (rexhealth.com)
  • This disorder has been called the postural tachycardia syndrome or POTS [ 1-3 ]. (uptodate.com)
  • Considering taking a vitamin or supplement to treat Postural+Tachycardia+Syndrome+(Pots)? (webmd.com)
  • Below is a list of common natural remedies used to treat or reduce the symptoms of Postural+Tachycardia+Syndrome+(Pots). (webmd.com)
  • Postural tachycardia syndrome (POTS) is a disabling condition that commonly affects otherwise normal young females. (nih.gov)
  • POT syndrome (POTS, postrual orthostatic tachycardia syndrome) is a nervous system disorder that causes lightheadedness and fainting when a person stands up. (medicinenet.com)
  • We report on a series of patients who developed autonomic dysfunction in the form of postural orthostatic tachycardia syndrome (POTS). (nih.gov)
  • Postural orthostatic tachycardia syndrome (POTS) is a disorder that can make someone feel faint or dizzy. (childrensmn.org)
  • The autonomic nervous system problems seen in POTS (also called postural tachycardia syndrome ) can affect children and adults. (childrensmn.org)
  • What Are the Signs & Symptoms of Postural Orthostatic Tachycardia Syndrome (POTS)? (childrensmn.org)
  • Who Gets Postural Orthostatic Tachycardia Syndrome (POTS)? (childrensmn.org)
  • How Is Postural Orthostatic Tachycardia Syndrome (POTS) Diagnosed? (childrensmn.org)
  • How Is Postural Orthostatic Tachycardia Syndrome (POTS) Treated? (childrensmn.org)
  • Postural orthostatic tachycardia syndrome (POTS) is a clinical entity characterized by orthostatic intolerance that includes an increase in heart rate of at least 30 bpm when one changes from a supine to an upright position within the first 10 minutes of standing without prolonged bed rest, medications, or other chronic debilitating disorders that impair autonomic reflexes. (aappublications.org)
  • A new scientific paper from Prof Newton's group (published this year in the Journal of Internal Medicine ) describes postural orthostatic tachycardia syndrome (POTS), an aspect of autonomic dysfunctionthat can produce substantial disability among otherwise healthy people. (prohealth.com)
  • The current study aimed to investigate the association between vitamin B 12 levels and postural orthostatic tachycardia syndrome (POTS) during adolescence when accelerated myelin synthesis increases the vitamin B 12 need. (aappublications.org)
  • Postural orthostatic tachycardia syndrome (POTS) is one of the most common forms of chronic orthostatic intolerance in the general population. (aappublications.org)
  • Postural (Orthostatic) Tachycardia Syndrome (POTS) is a topic covered in the 5-Minute Clinical Consult . (unboundmedicine.com)
  • It is uncertain whether deconditioning is a contributing cause or consequence of postural (orthostatic) tachycardia syndrome (POTS). (unboundmedicine.com)
  • However, tachycardia significantly increases the risk of stroke , sudden cardiac arrest, and death. (medicalnewstoday.com)
  • Catecholaminergic polymorphic ventricular tachycardia (CPVT) is an inherited arrhythmogenic disorder that causes syncopal episodes related with stress or emotion and even sudden cardiac deaths. (springer.com)
  • If CPVT is not recognized and treated, an episode of ventricular tachycardia may cause the heart to stop beating (cardiac arrest), leading to sudden death. (medlineplus.gov)
  • [2] In those in cardiac arrest due to ventricular tachycardia, cardiopulmonary resuscitation (CPR) and defibrillation is recommended. (rug.nl)
  • [2] Ventricular tachycardia may result in cardiac arrest and turn into ventricular fibrillation . (rug.nl)
  • Catecholaminergic polymorphic ventricular tachycardia (CPVT) is characterized by episodic syncope occurring during exercise or acute emotion in individuals without structural cardiac abnormalities. (nih.gov)
  • Treatment depends on the origin of the automatic tachycardia, however the mainstay of treatment is either antidysrhythmic medication or cardiac pacing. (wikipedia.org)
  • citation needed] Cardiac ectopy Clinical cardiac electrophysiology Electrical conduction system of the heart Supraventricular tachycardia Lister B et al. (wikipedia.org)
  • Diagnostic techniques such as continuous ambulatory electrocardiogram, intra-cardiac electrophysiology study (EPS), electrocardiogram (ECG), loop recorder, and blood tests are utilized for confirming ventricular tachycardia. (openpr.com)
  • Rising incidence of cardiac diseases such as cardiomyopathy, myocarditis, and valvular heart disease is anticipated to be the primary factor fueling the ventricular tachycardia market during the forecast period. (openpr.com)
  • A control module is coupled to the sensing module and the therapy delivery module and is configured to detect a tachycardia from the cardiac signal and initiate charging of the capacitor in response to detecting the tachycardia. (freepatentsonline.com)
  • 4. The method of claim 1, wherein determining if the ventricular tachycardia is monomorphic comprises comparing a first set of at least two cardiac signal waveforms to a second set of at least two cardiac signal waveforms to determine a morphology matching score without comparing the cardiac signal to a template determined before detecting the ventricular tachycardia. (freepatentsonline.com)
  • Pulseless ventricular tachycardia is a life-threatening cardiac emergency that ends in death without prompt and immediate treatment. (wisegeek.com)
  • As noted above, pulseless ventricular tachycardia is a temporary cardiac condition. (wisegeek.com)
  • Ventricular tachycardia (VT) in the presence of structural heart disease is associated with sudden cardiac death and warrants prompt attention. (ingentaconnect.com)
  • The purpose of this study is to test whether the addition of oral flecainide to standard therapy will reduce cardiac events compared to placebo plus standard therapy in patients with Catecholaminergic Polymorphic Ventricular Tachycardia. (bioportfolio.com)
  • Catecholaminergic polymorphic ventricular tachycardia (CPVT) is a highly lethal form of inherited primary electrical myocardial disease characterized by exercise- and stress-related adrenergic ventricular tachycardia without structural cardiac abnormalities. (genome.jp)
  • Mutations in calmodulin cause ventricular tachycardia and sudden cardiac death. (genome.jp)
  • In serious cases, Tachycardia can be life threatening and a sign of larger cardiac problems. (consumerinjurylawyers.com)
  • For this review, we searched the Ovid Medline database for presumptive CPVT cases using the keyword "catecholaminergic polymorphic ventricular tachycardia", covering the 60-year time period from 1950 to March 2009. (springer.com)
  • Ventricular tachycardia can occur due to coronary heart disease , aortic stenosis , cardiomyopathy , electrolyte problems (e.g., low blood levels of magnesium or potassium ), inherited channelopathies (e.g., long-QT syndrome ), catecholaminergic polymorphic ventricular tachycardia , arrhythmogenic right ventricular dysplasia , or a heart attack . (rug.nl)
  • Congenital problems include long QT syndrome and catecholaminergic polymorphic ventricular tachycardia . (rug.nl)
  • rhythm disorder that has proven amenable to modeling with iPSC-CMs is catecholaminergic polymorphic ventricular tachycardia (CPVT). (tripdatabase.com)
  • Catecholaminergic polymorphic ventricular tachycardia (CPVT) is a highly malignant form of arrhythmogenic disorder characterized by exercise- or emotional-induced polymorphic ventricular tachycardia in the absence of detectable structural heart disease. (nih.gov)
  • Catecholaminergic polymorphic ventricular tachycardia type 1 (CPVT1) predisposes to ventricular tachyarrhythmias (VTs) during high heart rates due to physical or psychological stress. (bioportfolio.com)
  • Implantable Cardioverter Defibrillator Use in Catecholaminergic Polymorphic Ventricular Tachycardia: A Systematic Review. (bioportfolio.com)
  • The implantable cardioverter defibrillator (ICD) may be associated with a high risk of complications in patients with catecholaminergic polymorphic ventricular tachycardia (CPVT). (bioportfolio.com)
  • Catecholaminergic polymorphic ventricular tachycardia patients with multiple genetic variants in the PACES CPVT Registry. (bioportfolio.com)
  • Catecholaminergic polymorphic ventricular tachycardia from bedside to bench and beyond. (genome.jp)
  • Genes, exercise and sudden death: molecular basis of familial catecholaminergic polymorphic ventricular tachycardia. (genome.jp)
  • Episodes of ventricular tachycardia can cause light-headedness, dizziness, and fainting (syncope). (medlineplus.gov)
  • Studies have shown dysfunction in the baroreflex mechanism and the autonomic nervous system, particularly in the sympathetic nervous system, in the pathophysiology of chronic fatigue syndrome, postural orthostatic tachycardia syndrome, and syncope. (aappublications.org)
  • Narrow complex tachycardias tend to originate in the atria, while wide complex tachycardias tend to originate in the ventricles. (wikipedia.org)
  • Supraventricular tachycardia is a series of rapid heartbeats that begin in or involve the upper chambers (atria) of the heart. (hopkinsmedicine.org)
  • Some patients have atrial tachycardia, a rapidly firing focus which may originate from either atria. (hopkinsmedicine.org)
  • A supraventricular tachycardia ( SVT ) is a tachycardia or rapid rhythm of the heart in which the origin of the electrical signal is either the atria or the AV node. (bionity.com)
  • This is in contrast to the deadlier ventricular tachycardias , which are rapid rhythms that originate from the ventricles of the heart, that is, below the atria or AV node. (bionity.com)
  • Still other people have an irritable group of cells in the atria that drives the tachycardia. (epnet.com)
  • In supraventricular tachycardia, the heart rate is sped up by an abnormal electrical impulse starting in the atria. (emedicinehealth.com)
  • Tachycardia may affect the upper or lower heart chambers, called the atria and ventricles. (biotronik.com)
  • As implied by the synonym junctional automatic tachycardia, the mechanism may be automaticity. (medscape.com)
  • When there is a problem with the electrical signals resulting in a faster-than-normal heartbeat, an individual has tachycardia. (medicalnewstoday.com)
  • Ventricular tachycardia (VT) is a rapid heartbeat that starts in the lower chambers of the heart (ventricles). (medlineplus.gov)
  • Tachycardia is the condition wherein the heartbeat of the person is more than normal. (buzzle.com)
  • As the heart rate increases in response to physical activity or emotional stress, it can trigger an abnormally fast heartbeat called ventricular tachycardia. (medlineplus.gov)
  • What happens when ventricular tachycardia throws off the steady, coordinated rhythm of your heartbeat? (mayoclinic.org)
  • Tachycardia (tak-ih-KAR-dee-uh) is an abnormally rapid heartbeat. (rchsd.org)
  • Radiofrequency ablation is necessary in tachycardias lasting more than 30 seconds. (medscape.com)
  • Ablation of the tachycardia was performed at the right angle intersection of lines drawn from the His and coronary sinus sites in the right lateral view. (medscape.com)
  • In this informative video, electrophysiologist Dr. Richard Hongo of San Francisco's CPMC explains signs, symptoms and treatment of ventricular tachycardia, using ablation - and helps you uinderstand if and when you should refer a patient for this procedure. (cpmc.org)
  • Fortunately, with the advent of radiofrequency ablation this form of tachycardia can be treated with high long-term success. (bmj.com)
  • The Role of Newer Technologies for Mapping and Ablation of Atrial Tachycardia. (wiley.com)
  • The BERLIN VT study is designed to evaluate the impact of prophylactic ventricular tachycardia (VT) ablation on all-cause mortality and unplanned hospital admission for congestive heart fa. (bioportfolio.com)
  • Radiofrequency ablation of idiopathic ventricular tachycardia. (biomedsearch.com)
  • Hyperadrenergic postural tachycardia syndrome in mast cell activation disorders. (nih.gov)
  • Also searched for Postural Tachycardia Syndrome . (clinicaltrials.gov)
  • Ventricular Tachycardia Industry Insight 2016 with Global API. (mynewsdesk.com)
  • The Report "Ventricular Tachycardia-Global API Manufacturers, Marketed and Phase III Drugs Landscape, 2016", provides comprehensive insights about the marketed drugs, drug sales, Phase III pipeline drugs and their API Manufacturers across the globe. (mynewsdesk.com)
  • Polymorphic ventricular tachycardia, on the other hand, is most commonly caused by abnormalities of ventricular muscle repolarization. (rug.nl)
  • The underlying cause of these episodes is the onset of fast ventricular tachycardia (bidirectional or polymorphic). (nih.gov)
  • Nevertheless, the management of highly symptomatic patients with focal atrial tachycardia can be problematic owing to the poor response to medical treatment. (bmj.com)
  • Focal atrial tachycardia (AT) accounts for up to 10% of supraventricular tachycardia. (bmj.com)
  • PAT is the most common form of tachycardia in infants and children. (healthline.com)
  • Supraventricular tachycardia or SVT is a type of heart rhythm disorder. (buzzle.com)
  • Atrial tachycardia is a heart rhythm with an elevated rate of electrical impulses being sent from the upper heart to the lower heart. (patientslikeme.com)
  • Atrial Tachycardia is a heart rhythm driven by pacemaker activity in atrial sites other than the SA node. (doctorslounge.com)
  • Supraventricular tachycardia (SVT) is defined as an abnormally rapid heart rhythm originating above the ventricles. (bioportfolio.com)
  • The origin or focus of the abnormal impulses distinguishes two types of atrial tachycardia depending upon whether the origin or focus of the abnormal impulse appears to involve a single (unifocal) site or multiple (multifocal) sites. (doctorslounge.com)
  • Treatments for tachycardia range from medication to surgery. (medtronic.com)
  • Data from patients with atrial tachycardia, who reported starting treatments within the last 5 years. (patientslikeme.com)
  • [2] The term ventricular tachycardias refers to the group of irregular heartbeats that includes ventricular tachycardia, ventricular fibrillation, and torsades de pointes . (rug.nl)
  • Supraventricular tachycardia does not include those tachycardia rhythms that originate from the ventricles (ventricular tachycardias) such as ventricular tachycardia or ventricular fibrillation. (emedicinehealth.com)
  • Ventricular tachycardia ( V-tach or VT ) is a type of regular, fast heart rate that arises from improper electrical activity in the ventricles of the heart . (rug.nl)
  • Damage to the ventricles can cause ventricular tachycardia. (denverhealth.org)
  • Tachycardia occurs when these beat too fast. (medicalnewstoday.com)
  • For patients who are at risk for certain forms of ventricular tachycardia, an implantable cardioverter defibrillator can quickly restore normal rhythm and prevent death if the dangerous rhythm occurs. (mayoclinic.org)
  • Atrial tachycardia occurs in some patients with myocardial diseases during systemic arterial hypoxia and in some patients with serious mitral valve disease. (doctorslounge.com)
  • Atrial tachycardia occurs when the atrial rate exceeds 100 bpm and the origin of electrical activity is within the atrium but outside the sinus node. (doctorslounge.com)
  • Supraventricular tachycardia often occurs in episodes with stretches of normal rhythm in between. (emedicinehealth.com)
  • However, if tachycardia occurs at rest, it may be a symptom of a serious health disorder. (biotronik.com)
  • Tachycardia occurs when the electrical impulses that coordinate heartbeats do not function properly and cause the heart to beat irregularly fast. (consumerinjurylawyers.com)
  • The Dysautonomia Information Network presents a film that provides answers, medical interviews, personal testimonies, facts and statistics about Postural Orthostatic Tachycardia Syndrome. (youtube.com)
  • Vitamin B 12 deficiency is associated with postural orthostatic tachycardia syndrome in adolescence. (aappublications.org)
  • The presence of a paced rhythm exactly at the upper rate limit with atrial sensing and exact A-V association warrants evaluation for pacemaker-mediated tachycardia (PMT). (medscape.com)
  • Pacemaker-mediated tachycardia: manufacturer specifics and spectrum of cases. (medscape.com)
  • Tracking of atrial flutter during DDD pacing: another form of pacemaker-mediated tachycardia. (medscape.com)
  • Validation of device algorithm to differentiate pacemaker-mediated tachycardia from tachycardia due to atrial tracking. (medscape.com)
  • Accuracy of the pacemaker-mediated tachycardia algorithm in Boston Scientific devices. (medscape.com)
  • Telemetered ECG tracing with surface lead II (top) and intracardiac electrograms (atrial electrogram [center] and ventricular electrogram [lower]) and marker channel (bottom) showing pacemaker-mediated tachycardia (PMT). (medscape.com)
  • Telemetered ECG tracing showing atrioventricular (AV)-paced rhythm at 60/min after termination of the pacemaker-mediated tachycardia (PMT). (medscape.com)
  • Electrocardiogram showing atrioventricular node re-entry tachycardia. (medscape.com)
  • This can lead to ventricular tachycardia. (medlineplus.gov)
  • Impaired calcium regulation in the heart can lead to ventricular tachycardia in people with CPVT. (medlineplus.gov)
  • However, factors such as high cost of therapy, low awareness among patients, and lack to access to medical facilities are restraining the ventricular tachycardia market in Middle East & Africa. (openpr.com)
  • What Are the Signs & Symptoms of Supraventricular Tachycardia (SVT)? (kidshealth.org)
  • In ventricular tachycardia, a misfire of your heart's electrical system throws the rhythm off. (mayoclinic.org)
  • Supraventricular tachycardia then is a rapid rhythm of the heart that begins in the upper chambers. (ahajournals.org)
  • It also can be due to an abnormal rhythm in the heart (called a dysrhythmia), such as supraventricular tachycardia (SVT). (rchsd.org)
  • Persistence of these tachycardias may lead to a cardiomyopathy and congestive heart failure. (medscape.com)
  • Ventricular tachycardia can occur due to coronary heart disease , aortic stenosis , cardiomyopathy , electrolyte problems , or a heart attack . (rug.nl)
  • Patients suffering from cardiomyopathy, myocarditis, or valvular heart disease are at a higher risk of developing ventricular tachycardia. (openpr.com)
  • If you have had a past heart attack, overactive thyroid, heart failure, blood chemistry imbalances or cardiomyopathy, your risk for developing tachycardia increases. (livestrong.com)
  • Tachycardia-Induced Cardiomyopathy. (wiley.com)
  • Ventricular tachycardia is the most common cause of death in people with cardiomyopathy. (rochester.edu)
  • A 7-year-old girl presented with a tachycardia-mediated cardiomyopathy secondary to a left atrial appendage tachycardia. (hindawi.com)
  • Tachycardia mediated cardiomyopathy can result as well. (hindawi.com)
  • Tachycardia-mediated cardiomyopathy was suspected. (hindawi.com)
  • Ventricular tachycardia (VT) is a fast, abnormal heart rate. (rochester.edu)
  • Some people with tachycardia may have no symptoms or complications. (medicalnewstoday.com)
  • When clinically applicable, the doctor may try to slow the rate, prevent subsequent episodes of tachycardia, and reduce risk complications. (medicalnewstoday.com)
  • Heart attack, stroke and death are some complications if tachycardia goes untreated. (livestrong.com)
  • A heart failure implantable cardioverter-defibrillator (ICD) delivers electroshocks to terminate tachycardias. (biotronik.com)