AnatomyOrganismsDiseasesChemicals and DrugsAnalytical, Diagnostic and Therapeutic Techniques and EquipmentPhenomena and ProcessesDisciplines and OccupationsAnthropology, Education, Sociology and Social PhenomenaTechnology, Industry, AgricultureInformation ScienceNamed GroupsHealth CareGeographicals
Streptococcus agalactiaeStreptococcusStreptococcal InfectionsStreptococcus pyogenesStreptococcus pneumoniaeStreptococcus mutansMastitis, BovineSerotypingBacterial ProteinsMastitisStreptococcus PhagesMeningitis, BacterialErythromycinStreptococcus suisDNA, BacterialAnti-Bacterial AgentsStreptococcus bovisBacterial AdhesionVaginaStreptococcus mitisBacterial CapsulesMicrobial Sensitivity TestsStreptococcus equiGenes, BacterialMolecular Sequence DataStreptococcus oralisDrug Resistance, BacterialVirulenceProphagesAntigens, BacterialStreptococcus sobrinusAminoacyltransferasesSequence Analysis, DNABacterial Typing TechniquesLincosamidesEnterococcus faecalisGram-Positive BacteriaBacteriological TechniquesCulture MediaClindamycinVirulence FactorsCattleKeratin-4Adhesins, BacterialPneumococcal InfectionsStreptococcus gordoniiGene Expression Regulation, BacterialElectrophoresis, Gel, Pulsed-FieldTeichoic AcidsPyometraAmobarbitalTetracycline ResistanceRandom Amplified Polymorphic DNA TechniqueStreptococcus thermophilusGenome, BacterialHemolysin ProteinsInfant, NewbornPolysaccharides, BacterialFasciitis, NecrotizingLactoperoxidaseArcanobacteriumPolymerase Chain ReactionMacrolidesMilkDNA FingerprintingProkaryotic Initiation Factor-2Carrier StateMycoplasma InfectionsReceptors, FibrinogenAntibodies, BacterialInterspersed Repetitive SequencesDNA Transposable ElementsMeningitisPhylogenyBase SequencePolysaccharide-LyasesGenetic VariationAmino Acid SequenceMolecular TypingZimbabwePigments, BiologicalPregnancy Complications, InfectiousTetracyclineDrug Resistance, MicrobialBacteriaDairyingArthritis, InfectiousStaphylococcusMultilocus Sequence TypingCell WallGene OrderMouthStaphylococcus aureusHemolysisColony Count, MicrobialGenotypeStreptolysinsRectumPregnancyFimbriae, BacterialSpecies SpecificityEnterococcusCattle DiseasesGram-Negative BacteriaAnti-Infective AgentsStreptococcus intermediusGenomic IslandsConjugation, GeneticPharyngitisGene Transfer, HorizontalBloodDental PlaqueBacteremiaPneumonia, PneumococcalMolecular EpidemiologyCluster AnalysisMycoplasmaFibrinogenStreptococcal VaccinesRNA, BacterialMutagenesis, InsertionalGene DeletionEscherichia coliPenicillin ResistanceFimbriae ProteinsCloning, MolecularSheep DiseasesOperonGram-Positive Bacterial InfectionsSalivaPharynx
Streptococcus agalactiae: A bacterium which causes mastitis in cattle and occasionally in man.Streptococcus: A genus of gram-positive, coccoid bacteria whose organisms occur in pairs or chains. No endospores are produced. Many species exist as commensals or parasites on man or animals with some being highly pathogenic. A few species are saprophytes and occur in the natural environment.Streptococcal Infections: Infections with bacteria of the genus STREPTOCOCCUS.Streptococcus pyogenes: A species of gram-positive, coccoid bacteria isolated from skin lesions, blood, inflammatory exudates, and the upper respiratory tract of humans. It is a group A hemolytic Streptococcus that can cause SCARLET FEVER and RHEUMATIC FEVER.Streptococcus pneumoniae: A gram-positive organism found in the upper respiratory tract, inflammatory exudates, and various body fluids of normal and/or diseased humans and, rarely, domestic animals.Streptococcus mutans: A polysaccharide-producing species of STREPTOCOCCUS isolated from human dental plaque.Mastitis, Bovine: INFLAMMATION of the UDDER in cows.Serotyping: Process of determining and distinguishing species of bacteria or viruses based on antigens they share.Bacterial Proteins: Proteins found in any species of bacterium.Mastitis: INFLAMMATION of the BREAST, or MAMMARY GLAND.Streptococcus Phages: Viruses whose host is Streptococcus.Meningitis, Bacterial: Bacterial infections of the leptomeninges and subarachnoid space, frequently involving the cerebral cortex, cranial nerves, cerebral blood vessels, spinal cord, and nerve roots.Erythromycin: A bacteriostatic antibiotic macrolide produced by Streptomyces erythreus. Erythromycin A is considered its major active component. In sensitive organisms, it inhibits protein synthesis by binding to 50S ribosomal subunits. This binding process inhibits peptidyl transferase activity and interferes with translocation of amino acids during translation and assembly of proteins.Streptococcus suis: A species of STREPTOCOCCUS isolated from pigs. It is a pathogen of swine but rarely occurs in humans.DNA, Bacterial: Deoxyribonucleic acid that makes up the genetic material of bacteria.Anti-Bacterial Agents: Substances that reduce the growth or reproduction of BACTERIA.Streptococcus bovis: A species of gram-positive, coccoid bacteria commonly found in the alimentary tract of cows, sheep, and other ruminants. It occasionally is encountered in cases of human endocarditis. This species is nonhemolytic.Bacterial Adhesion: Physicochemical property of fimbriated (FIMBRIAE, BACTERIAL) and non-fimbriated bacteria of attaching to cells, tissue, and nonbiological surfaces. It is a factor in bacterial colonization and pathogenicity.Vagina: The genital canal in the female, extending from the UTERUS to the VULVA. (Stedman, 25th ed)Streptococcus mitis: A species of gram-positive, coccoid bacteria commensal in the respiratory tract.Bacterial Capsules: An envelope of loose gel surrounding a bacterial cell which is associated with the virulence of pathogenic bacteria. Some capsules have a well-defined border, whereas others form a slime layer that trails off into the medium. Most capsules consist of relatively simple polysaccharides but there are some bacteria whose capsules are made of polypeptides.Microbial Sensitivity Tests: Any tests that demonstrate the relative efficacy of different chemotherapeutic agents against specific microorganisms (i.e., bacteria, fungi, viruses).Streptococcus equi: A species of gram-positive, coccoid bacteria isolated from abscesses in submaxillary glands and mucopurulent discharges of the upper respiratory tract of horses. This organism belongs to Group C streptococci with regards to antigen response and is known to cause strangles. The subspecies S. zooepidemicus is also considered a pathogen of horses.Genes, Bacterial: The functional hereditary units of BACTERIA.Molecular Sequence Data: Descriptions of specific amino acid, carbohydrate, or nucleotide sequences which have appeared in the published literature and/or are deposited in and maintained by databanks such as GENBANK, European Molecular Biology Laboratory (EMBL), National Biomedical Research Foundation (NBRF), or other sequence repositories.Streptococcus oralis: A species of gram-positive, coccoid bacteria that is numerous in the mouth and throat. It is a common cause of endocarditis and is also implicated in dental plaque formation.Drug Resistance, Bacterial: The ability of bacteria to resist or to become tolerant to chemotherapeutic agents, antimicrobial agents, or antibiotics. This resistance may be acquired through gene mutation or foreign DNA in transmissible plasmids (R FACTORS).Virulence: The degree of pathogenicity within a group or species of microorganisms or viruses as indicated by case fatality rates and/or the ability of the organism to invade the tissues of the host. The pathogenic capacity of an organism is determined by its VIRULENCE FACTORS.Prophages: Genomes of temperate BACTERIOPHAGES integrated into the DNA of their bacterial host cell. The prophages can be duplicated for many cell generations until some stimulus induces its activation and virulence.Antigens, Bacterial: Substances elaborated by bacteria that have antigenic activity.Streptococcus sobrinus: A species of gram-positive, coccoid bacteria isolated from the human tooth surface. Strains have been shown to be cariogenic in experimental animals and may be associated with human dental caries.Aminoacyltransferases: Enzymes that catalyze the transfer of an aminoacyl group from donor to acceptor resulting in the formation of an ester or amide linkage. EC 2.3.2.Sequence Analysis, DNA: A multistage process that includes cloning, physical mapping, subcloning, determination of the DNA SEQUENCE, and information analysis.Bacterial Typing Techniques: Procedures for identifying types and strains of bacteria. The most frequently employed typing systems are BACTERIOPHAGE TYPING and SEROTYPING as well as bacteriocin typing and biotyping.Lincosamides: A family of LINCOMYCIN-related glycosides that contain a pyrrolidine ring linked via an amide-bond to a pyranose moiety. Individual members of this family are defined by the arrangement of specific constituent groups on the lyncomycin molecule. Many lincosamides are ANTIBIOTICS produced by a variety STREPTOMYCES species.Enterococcus faecalis: A species of gram-positive, coccoid bacteria commonly isolated from clinical specimens and the human intestinal tract. Most strains are nonhemolytic.Gram-Positive Bacteria: Bacteria which retain the crystal violet stain when treated by Gram's method.Bacteriological Techniques: Techniques used in studying bacteria.Culture Media: Any liquid or solid preparation made specifically for the growth, storage, or transport of microorganisms or other types of cells. The variety of media that exist allow for the culturing of specific microorganisms and cell types, such as differential media, selective media, test media, and defined media. Solid media consist of liquid media that have been solidified with an agent such as AGAR or GELATIN.Clindamycin: An antibacterial agent that is a semisynthetic analog of LINCOMYCIN.Virulence Factors: Those components of an organism that determine its capacity to cause disease but are not required for its viability per se. Two classes have been characterized: TOXINS, BIOLOGICAL and surface adhesion molecules that effect the ability of the microorganism to invade and colonize a host. (From Davis et al., Microbiology, 4th ed. p486)Cattle: Domesticated bovine animals of the genus Bos, usually kept on a farm or ranch and used for the production of meat or dairy products or for heavy labor.Keratin-4: A type II keratin that is found associated with the KERATIN-13 in the internal stratified EPITHELIUM. Defects in gene for keratin-4 are a cause of HEREDITARY MUCOSAL LEUKOKERATOSIS.Adhesins, Bacterial: Cell-surface components or appendages of bacteria that facilitate adhesion (BACTERIAL ADHESION) to other cells or to inanimate surfaces. Most fimbriae (FIMBRIAE, BACTERIAL) of gram-negative bacteria function as adhesins, but in many cases it is a minor subunit protein at the tip of the fimbriae that is the actual adhesin. In gram-positive bacteria, a protein or polysaccharide surface layer serves as the specific adhesin. What is sometimes called polymeric adhesin (BIOFILMS) is distinct from protein adhesin.Pneumococcal Infections: Infections with bacteria of the species STREPTOCOCCUS PNEUMONIAE.Streptococcus gordonii: A species of gram-positive, facultatively anaerobic bacteria in the family STREPTOCOCCACEAE. It is a normal inhabitant of the human oral cavity, and causes DENTAL PLAQUE and ENDOCARDITIS. It is being investigated as a vehicle for vaccine delivery.Gene Expression Regulation, Bacterial: Any of the processes by which cytoplasmic or intercellular factors influence the differential control of gene action in bacteria.Electrophoresis, Gel, Pulsed-Field: Gel electrophoresis in which the direction of the electric field is changed periodically. This technique is similar to other electrophoretic methods normally used to separate double-stranded DNA molecules ranging in size up to tens of thousands of base-pairs. However, by alternating the electric field direction one is able to separate DNA molecules up to several million base-pairs in length.Teichoic Acids: Bacterial polysaccharides that are rich in phosphodiester linkages. They are the major components of the cell walls and membranes of many bacteria.Pyometra: An accumulation of PUS in the uterine cavity (UTERUS). Pyometra generally indicates the presence of infections.Amobarbital: A barbiturate with hypnotic and sedative properties (but not antianxiety). Adverse effects are mainly a consequence of dose-related CNS depression and the risk of dependence with continued use is high. (From Martindale, The Extra Pharmacopoeia, 30th ed, p565)Tetracycline Resistance: Nonsusceptibility of bacteria to the action of TETRACYCLINE which inhibits aminoacyl-tRNA binding to the 30S ribosomal subunit during protein synthesis.Random Amplified Polymorphic DNA Technique: Technique that utilizes low-stringency polymerase chain reaction (PCR) amplification with single primers of arbitrary sequence to generate strain-specific arrays of anonymous DNA fragments. RAPD technique may be used to determine taxonomic identity, assess kinship relationships, analyze mixed genome samples, and create specific probes.Streptococcus thermophilus: A species of thermophilic, gram-positive bacteria found in MILK and milk products.Genome, Bacterial: The genetic complement of a BACTERIA as represented in its DNA.Hemolysin Proteins: Proteins from BACTERIA and FUNGI that are soluble enough to be secreted to target ERYTHROCYTES and insert into the membrane to form beta-barrel pores. Biosynthesis may be regulated by HEMOLYSIN FACTORS.Infant, Newborn: An infant during the first month after birth.Polysaccharides, Bacterial: Polysaccharides found in bacteria and in capsules thereof.Fasciitis, Necrotizing: A fulminating bacterial infection of the deep layers of the skin and FASCIA. It can be caused by many different organisms, with STREPTOCOCCUS PYOGENES being the most common.Lactoperoxidase: An enzyme derived from cow's milk. It catalyzes the radioiodination of tyrosine and its derivatives and of peptides containing tyrosine.Arcanobacterium: A genus of facultatively anaerobic, gram-positive bacteria in the family ACTINOMYCETACEAE, order ACTINOMYCETALES. They are obligate parasites of the PHARYNX in humans and farm animals.Polymerase Chain Reaction: In vitro method for producing large amounts of specific DNA or RNA fragments of defined length and sequence from small amounts of short oligonucleotide flanking sequences (primers). The essential steps include thermal denaturation of the double-stranded target molecules, annealing of the primers to their complementary sequences, and extension of the annealed primers by enzymatic synthesis with DNA polymerase. The reaction is efficient, specific, and extremely sensitive. Uses for the reaction include disease diagnosis, detection of difficult-to-isolate pathogens, mutation analysis, genetic testing, DNA sequencing, and analyzing evolutionary relationships.Macrolides: A group of often glycosylated macrocyclic compounds formed by chain extension of multiple PROPIONATES cyclized into a large (typically 12, 14, or 16)-membered lactone. Macrolides belong to the POLYKETIDES class of natural products, and many members exhibit ANTIBIOTIC properties.Milk: The white liquid secreted by the mammary glands. It contains proteins, sugar, lipids, vitamins, and minerals.DNA Fingerprinting: A technique for identifying individuals of a species that is based on the uniqueness of their DNA sequence. Uniqueness is determined by identifying which combination of allelic variations occur in the individual at a statistically relevant number of different loci. In forensic studies, RESTRICTION FRAGMENT LENGTH POLYMORPHISM of multiple, highly polymorphic VNTR LOCI or MICROSATELLITE REPEAT loci are analyzed. The number of loci used for the profile depends on the ALLELE FREQUENCY in the population.Prokaryotic Initiation Factor-2: The largest of the three prokaryotic initiation factors with a molecular size of approximately 80 kD. It functions in the transcription initiation process by promoting the binding of formylmethionine-tRNA to the P-site of the 30S ribosome and by preventing the incorrect binding of elongator tRNA to the translation initiation site.Carrier State: The condition of harboring an infective organism without manifesting symptoms of infection. The organism must be readily transmissible to another susceptible host.Mycoplasma Infections: Infections with species of the genus MYCOPLASMA.Receptors, Fibrinogen: Receptors that bind FIBRINOGEN through distinct adhesive sequences on the fibrinogen molecule. Although MACROPHAGE-1 ANTIGEN is considered an important signaling molecule for fibrinogen interaction, a variety of INTEGRINS from all three major families, (beta1, beta2, and beta3) have been shown to bind fibrinogen.Antibodies, Bacterial: Immunoglobulins produced in a response to BACTERIAL ANTIGENS.Interspersed Repetitive Sequences: Copies of transposable elements interspersed throughout the genome, some of which are still active and often referred to as "jumping genes". There are two classes of interspersed repetitive elements. Class I elements (or RETROELEMENTS - such as retrotransposons, retroviruses, LONG INTERSPERSED NUCLEOTIDE ELEMENTS and SHORT INTERSPERSED NUCLEOTIDE ELEMENTS) transpose via reverse transcription of an RNA intermediate. Class II elements (or DNA TRANSPOSABLE ELEMENTS - such as transposons, Tn elements, insertion sequence elements and mobile gene cassettes of bacterial integrons) transpose directly from one site in the DNA to another.DNA Transposable Elements: Discrete segments of DNA which can excise and reintegrate to another site in the genome. Most are inactive, i.e., have not been found to exist outside the integrated state. DNA transposable elements include bacterial IS (insertion sequence) elements, Tn elements, the maize controlling elements Ac and Ds, Drosophila P, gypsy, and pogo elements, the human Tigger elements and the Tc and mariner elements which are found throughout the animal kingdom.Meningitis: Inflammation of the coverings of the brain and/or spinal cord, which consist of the PIA MATER; ARACHNOID; and DURA MATER. Infections (viral, bacterial, and fungal) are the most common causes of this condition, but subarachnoid hemorrhage (HEMORRHAGES, SUBARACHNOID), chemical irritation (chemical MENINGITIS), granulomatous conditions, neoplastic conditions (CARCINOMATOUS MENINGITIS), and other inflammatory conditions may produce this syndrome. (From Joynt, Clinical Neurology, 1994, Ch24, p6)Phylogeny: The relationships of groups of organisms as reflected by their genetic makeup.Base Sequence: The sequence of PURINES and PYRIMIDINES in nucleic acids and polynucleotides. It is also called nucleotide sequence.Polysaccharide-Lyases: A group of carbon-oxygen lyases. These enzymes catalyze the breakage of a carbon-oxygen bond in polysaccharides leading to an unsaturated product and the elimination of an alcohol. EC 4.2.2.Genetic Variation: Genotypic differences observed among individuals in a population.Amino Acid Sequence: The order of amino acids as they occur in a polypeptide chain. This is referred to as the primary structure of proteins. It is of fundamental importance in determining PROTEIN CONFORMATION.Molecular Typing: Using MOLECULAR BIOLOGY techniques, such as DNA SEQUENCE ANALYSIS; PULSED-FIELD GEL ELECTROPHORESIS; and DNA FINGERPRINTING, to identify, classify, and compare organisms and their subtypes.Zimbabwe: A republic in southern Africa, east of ZAMBIA and BOTSWANA and west of MOZAMBIQUE. Its capital is Harare. It was formerly called Rhodesia and Southern Rhodesia.Pigments, Biological: Any normal or abnormal coloring matter in PLANTS; ANIMALS or micro-organisms.Pregnancy Complications, Infectious: The co-occurrence of pregnancy and an INFECTION. The infection may precede or follow FERTILIZATION.Tetracycline: A naphthacene antibiotic that inhibits AMINO ACYL TRNA binding during protein synthesis.Drug Resistance, Microbial: The ability of microorganisms, especially bacteria, to resist or to become tolerant to chemotherapeutic agents, antimicrobial agents, or antibiotics. This resistance may be acquired through gene mutation or foreign DNA in transmissible plasmids (R FACTORS).Bacteria: One of the three domains of life (the others being Eukarya and ARCHAEA), also called Eubacteria. They are unicellular prokaryotic microorganisms which generally possess rigid cell walls, multiply by cell division, and exhibit three principal forms: round or coccal, rodlike or bacillary, and spiral or spirochetal. Bacteria can be classified by their response to OXYGEN: aerobic, anaerobic, or facultatively anaerobic; by the mode by which they obtain their energy: chemotrophy (via chemical reaction) or PHOTOTROPHY (via light reaction); for chemotrophs by their source of chemical energy: CHEMOLITHOTROPHY (from inorganic compounds) or chemoorganotrophy (from organic compounds); and by their source for CARBON; NITROGEN; etc.; HETEROTROPHY (from organic sources) or AUTOTROPHY (from CARBON DIOXIDE). They can also be classified by whether or not they stain (based on the structure of their CELL WALLS) with CRYSTAL VIOLET dye: gram-negative or gram-positive.DairyingArthritis, Infectious: Arthritis caused by BACTERIA; RICKETTSIA; MYCOPLASMA; VIRUSES; FUNGI; or PARASITES.Staphylococcus: A genus of gram-positive, facultatively anaerobic, coccoid bacteria. Its organisms occur singly, in pairs, and in tetrads and characteristically divide in more than one plane to form irregular clusters. Natural populations of Staphylococcus are found on the skin and mucous membranes of warm-blooded animals. Some species are opportunistic pathogens of humans and animals.Multilocus Sequence Typing: Direct nucleotide sequencing of gene fragments from multiple housekeeping genes for the purpose of phylogenetic analysis, organism identification, and typing of species, strain, serovar, or other distinguishable phylogenetic level.Cell Wall: The outermost layer of a cell in most PLANTS; BACTERIA; FUNGI; and ALGAE. The cell wall is usually a rigid structure that lies external to the CELL MEMBRANE, and provides a protective barrier against physical or chemical agents.Gene Order: The sequential location of genes on a chromosome.Mouth: The oval-shaped oral cavity located at the apex of the digestive tract and consisting of two parts: the vestibule and the oral cavity proper.Staphylococcus aureus: Potentially pathogenic bacteria found in nasal membranes, skin, hair follicles, and perineum of warm-blooded animals. They may cause a wide range of infections and intoxications.Hemolysis: The destruction of ERYTHROCYTES by many different causal agents such as antibodies, bacteria, chemicals, temperature, and changes in tonicity.Colony Count, Microbial: Enumeration by direct count of viable, isolated bacterial, archaeal, or fungal CELLS or SPORES capable of growth on solid CULTURE MEDIA. The method is used routinely by environmental microbiologists for quantifying organisms in AIR; FOOD; and WATER; by clinicians for measuring patients' microbial load; and in antimicrobial drug testing.Genotype: The genetic constitution of the individual, comprising the ALLELES present at each GENETIC LOCUS.Streptolysins: Exotoxins produced by certain strains of streptococci, particularly those of group A (STREPTOCOCCUS PYOGENES), that cause HEMOLYSIS.Rectum: The distal segment of the LARGE INTESTINE, between the SIGMOID COLON and the ANAL CANAL.Pregnancy: The status during which female mammals carry their developing young (EMBRYOS or FETUSES) in utero before birth, beginning from FERTILIZATION to BIRTH.Fimbriae, Bacterial: Thin, hairlike appendages, 1 to 20 microns in length and often occurring in large numbers, present on the cells of gram-negative bacteria, particularly Enterobacteriaceae and Neisseria. Unlike flagella, they do not possess motility, but being protein (pilin) in nature, they possess antigenic and hemagglutinating properties. They are of medical importance because some fimbriae mediate the attachment of bacteria to cells via adhesins (ADHESINS, BACTERIAL). Bacterial fimbriae refer to common pili, to be distinguished from the preferred use of "pili", which is confined to sex pili (PILI, SEX).Species Specificity: The restriction of a characteristic behavior, anatomical structure or physical system, such as immune response; metabolic response, or gene or gene variant to the members of one species. It refers to that property which differentiates one species from another but it is also used for phylogenetic levels higher or lower than the species.Enterococcus: A genus of gram-positive, coccoid bacteria consisting of organisms causing variable hemolysis that are normal flora of the intestinal tract. Previously thought to be a member of the genus STREPTOCOCCUS, it is now recognized as a separate genus.Cattle Diseases: Diseases of domestic cattle of the genus Bos. It includes diseases of cows, yaks, and zebus.Gram-Negative Bacteria: Bacteria which lose crystal violet stain but are stained pink when treated by Gram's method.Anti-Infective Agents: Substances that prevent infectious agents or organisms from spreading or kill infectious agents in order to prevent the spread of infection.Streptococcus intermedius: A species of gram-positive bacteria in the STREPTOCOCCUS MILLERI GROUP. It is commonly found in the oropharynx flora and has a proclivity for abscess formation, most characteristically in the CENTRAL NERVOUS SYSTEM and LIVER.Genomic Islands: Distinct units in some bacterial, bacteriophage or plasmid GENOMES that are types of MOBILE GENETIC ELEMENTS. Encoded in them are a variety of fitness conferring genes, such as VIRULENCE FACTORS (in "pathogenicity islands or islets"), ANTIBIOTIC RESISTANCE genes, or genes required for SYMBIOSIS (in "symbiosis islands or islets"). They range in size from 10 - 500 kilobases, and their GC CONTENT and CODON usage differ from the rest of the genome. They typically contain an INTEGRASE gene, although in some cases this gene has been deleted resulting in "anchored genomic islands".Conjugation, Genetic: A parasexual process in BACTERIA; ALGAE; FUNGI; and ciliate EUKARYOTA for achieving exchange of chromosome material during fusion of two cells. In bacteria, this is a uni-directional transfer of genetic material; in protozoa it is a bi-directional exchange. In algae and fungi, it is a form of sexual reproduction, with the union of male and female gametes.Pharyngitis: Inflammation of the throat (PHARYNX).Gene Transfer, Horizontal: The naturally occurring transmission of genetic information between organisms, related or unrelated, circumventing parent-to-offspring transmission. Horizontal gene transfer may occur via a variety of naturally occurring processes such as GENETIC CONJUGATION; GENETIC TRANSDUCTION; and TRANSFECTION. It may result in a change of the recipient organism's genetic composition (TRANSFORMATION, GENETIC).Blood: The body fluid that circulates in the vascular system (BLOOD VESSELS). Whole blood includes PLASMA and BLOOD CELLS.Dental Plaque: A film that attaches to teeth, often causing DENTAL CARIES and GINGIVITIS. It is composed of MUCINS, secreted from salivary glands, and microorganisms.Bacteremia: The presence of viable bacteria circulating in the blood. Fever, chills, tachycardia, and tachypnea are common acute manifestations of bacteremia. The majority of cases are seen in already hospitalized patients, most of whom have underlying diseases or procedures which render their bloodstreams susceptible to invasion.Pneumonia, Pneumococcal: A febrile disease caused by STREPTOCOCCUS PNEUMONIAE.Molecular Epidemiology: The application of molecular biology to the answering of epidemiological questions. The examination of patterns of changes in DNA to implicate particular carcinogens and the use of molecular markers to predict which individuals are at highest risk for a disease are common examples.Cluster Analysis: A set of statistical methods used to group variables or observations into strongly inter-related subgroups. In epidemiology, it may be used to analyze a closely grouped series of events or cases of disease or other health-related phenomenon with well-defined distribution patterns in relation to time or place or both.Mycoplasma: A genus of gram-negative, mostly facultatively anaerobic bacteria in the family MYCOPLASMATACEAE. The cells are bounded by a PLASMA MEMBRANE and lack a true CELL WALL. Its organisms are pathogens found on the MUCOUS MEMBRANES of humans, ANIMALS, and BIRDS.Fibrinogen: Plasma glycoprotein clotted by thrombin, composed of a dimer of three non-identical pairs of polypeptide chains (alpha, beta, gamma) held together by disulfide bonds. Fibrinogen clotting is a sol-gel change involving complex molecular arrangements: whereas fibrinogen is cleaved by thrombin to form polypeptides A and B, the proteolytic action of other enzymes yields different fibrinogen degradation products.Streptococcal Vaccines: Vaccines or candidate vaccines used to prevent STREPTOCOCCAL INFECTIONS.RNA, Bacterial: Ribonucleic acid in bacteria having regulatory and catalytic roles as well as involvement in protein synthesis.Mutagenesis, Insertional: Mutagenesis where the mutation is caused by the introduction of foreign DNA sequences into a gene or extragenic sequence. This may occur spontaneously in vivo or be experimentally induced in vivo or in vitro. Proviral DNA insertions into or adjacent to a cellular proto-oncogene can interrupt GENETIC TRANSLATION of the coding sequences or interfere with recognition of regulatory elements and cause unregulated expression of the proto-oncogene resulting in tumor formation.Gene Deletion: A genetic rearrangement through loss of segments of DNA or RNA, bringing sequences which are normally separated into close proximity. This deletion may be detected using cytogenetic techniques and can also be inferred from the phenotype, indicating a deletion at one specific locus.Escherichia coli: A species of gram-negative, facultatively anaerobic, rod-shaped bacteria (GRAM-NEGATIVE FACULTATIVELY ANAEROBIC RODS) commonly found in the lower part of the intestine of warm-blooded animals. It is usually nonpathogenic, but some strains are known to produce DIARRHEA and pyogenic infections. Pathogenic strains (virotypes) are classified by their specific pathogenic mechanisms such as toxins (ENTEROTOXIGENIC ESCHERICHIA COLI), etc.Penicillin Resistance: Nonsusceptibility of an organism to the action of penicillins.Fimbriae Proteins: Proteins that are structural components of bacterial fimbriae (FIMBRIAE, BACTERIAL) or sex pili (PILI, SEX).Cloning, Molecular: The insertion of recombinant DNA molecules from prokaryotic and/or eukaryotic sources into a replicating vehicle, such as a plasmid or virus vector, and the introduction of the resultant hybrid molecules into recipient cells without altering the viability of those cells.Sheep Diseases: Diseases of domestic and mountain sheep of the genus Ovis.Operon: In bacteria, a group of metabolically related genes, with a common promoter, whose transcription into a single polycistronic MESSENGER RNA is under the control of an OPERATOR REGION.Gram-Positive Bacterial Infections: Infections caused by bacteria that retain the crystal violet stain (positive) when treated by the gram-staining method.Saliva: The clear, viscous fluid secreted by the SALIVARY GLANDS and mucous glands of the mouth. It contains MUCINS, water, organic salts, and ptylin.Pharynx: A funnel-shaped fibromuscular tube that conducts food to the ESOPHAGUS, and air to the LARYNX and LUNGS. It is located posterior to the NASAL CAVITY; ORAL CAVITY; and LARYNX, and extends from the SKULL BASE to the inferior border of the CRICOID CARTILAGE anteriorly and to the inferior border of the C6 vertebra posteriorly. It is divided into the NASOPHARYNX; OROPHARYNX; and HYPOPHARYNX (laryngopharynx).

GM-CSF-deficient mice are susceptible to pulmonary group B streptococcal infection. (1/1478)

Granulocyte-macrophage colony-stimulating factor (GM-CSF) gene-targeted mice (GM-/-) cleared group B streptococcus (GBS) from the lungs more slowly than wild-type mice. Expression of GM-CSF in the respiratory epithelium of GM-/- mice improved bacterial clearance to levels greater than that in wild-type GM+/+ mice. Acute aerosolization of GM-CSF to GM+/+ mice significantly enhanced clearance of GBS at 24 hours. GBS infection was associated with increased neutrophilic infiltration in lungs of GM-/- mice, while macrophage infiltrates predominated in wild-type mice, suggesting an abnormality in macrophage clearance of bacteria in the absence of GM-CSF. While phagocytosis of GBS was unaltered, production of superoxide radicals and hydrogen peroxide was markedly deficient in macrophages from GM-/- mice. Lipid peroxidation, assessed by measuring the isoprostane 8-iso-PGF2alpha, was decreased in the lungs of GM-/- mice. GM-CSF plays an important role in GBS clearance in vivo, mediated in part by its role in enhancing superoxide and hydrogen peroxide production and bacterial killing by alveolar macrophages.  (+info)

Maternal immunization. (2/1478)

Maternal immunization can enhance passive immunity of infants to pathogens that cause life-threatening illnesses. In most instances, immunization during pregnancy will provide important protection for the woman as well as for her offspring. The tetanus toxoid and influenza vaccines are examples of vaccines that provide a double benefit. Other vaccines under evaluation include those for respiratory syncytial virus, pneumococci, group B streptococci, and Haemophilus influenzae type b. Although most IgG antibody crosses the placenta in the third trimester, the process is time-dependent, dictating that immunization should be accomplished ideally at least 6 weeks prior to delivery. IgG1 antibodies are transferred preferentially. Maternal immunization has not interfered with active immunization of the infant. Inactivated vaccines administered in the third trimester of pregnancy pose no known risk to the woman or to her fetus.  (+info)

Serotypes VI and VIII predominate among group B streptococci isolated from pregnant Japanese women. (3/1478)

Infection by group B streptococcus (GBS) is an important cause of bacterial disease in neonates, pregnant women, and nonpregnant adults. Whereas serotypes Ia, Ib, II, III, and V are most commonly associated with colonization and disease in the United States, strains of other serotypes have been isolated from patients in Japan. By use of an inhibition ELISA, the serotypes of 73 vaginal colonizing GBS strains isolated from healthy pregnant Japanese women were investigated. Twenty-six (35.6%) were type VIII, 18 (24.7%) were type VI, and the remaining 29 were distributed among more traditional serotypes. Strains were also tested by immunoblot for the presence of GBS surface proteins. Fifty-three (72.6%) of the 73 strains expressed one or more laddering GBS proteins. These data show that type VI and VIII GBS strains are common vaginal isolates in pregnant Japanese women and that one or more laddering proteins are present in most GBS strains.  (+info)

Capsular sialic acid limits C5a production on type III group B streptococci. (4/1478)

The majority of type III group B streptococcus (GBS) human neonatal infections are caused by a genetically related subgroup called III-3. We have proposed that a bacterial enzyme, C5a-ase, contributes to the pathogenesis of neonatal infections with GBS by rapidly inactivating C5a, a potent pro-inflammatory molecule, but many III-3 strains do not express C5a-ase. The amount of C5a produced in serum following incubation with representative type III strains was quantitated in order to better understand the relationship between C5a production and C5a-ase expression. C5a production following incubation of bacteria with serum depleted of antibody to the bacterial surface was inversely proportional to the sialic acid content of the bacterial capsule, with the more heavily sialylated III-3 strains generating less C5a than the less-virulent, less-sialylated III-2 strains. The amount of C5a produced correlated significantly with C3 deposition on each bacterial strain. Repletion with type-specific antibody caused increased C3b deposition and C5a production through alternative pathway activation, but C5a was functionally inactivated by strains that expressed C5a-ase. The increased virulence of III-3 strains compared to that of III-2 strains results at least partially from the higher sialic acid content of III-3 strains, which inhibits both opsonophagocytic killing and C5a production in the absence of type-specific antibody. We propose that C5a-ase is not necessary for III-3 strains to cause invasive disease because the high sialic acid content of III-3 strains inhibits C5a production.  (+info)

Presence of mefA and mefE genes in Streptococcus agalactiae. (5/1478)

Eighteen unrelated clinical isolates of Streptococcus agalactiae with the M phenotype harbored an mef gene. DNA sequencing showed that one of nine strains contained mefA (producing one amino acid substitution), whereas the remaining eight carried mefE (identity, 100%). Restriction analysis of PCR products indicated that the nine other strains also contained mefE.  (+info)

Role of antibody and complement in opsonization of group B streptococci. (6/1478)

A requirement for the classic complement pathway in opsonization of group B streptococci was observed by using both a chemiluminescence and a radiolabeled bacterial uptake technique. The classic pathway increased levels of opsonization for types Ia and II stock and wild strains and for some type III wild strains. In contrast, other type III wild strains and the type III stock strain had accelerated kinetics of uptake in the presence of an intact classic pathway, but the level of opsonization was unchanged from that with antibody alone. We could not demonstrate a significant role for the alternative pathway in opsonizing stock or wild strains of group B streptococci. Futhermore, electrophoretic and complement consumption analysis by hemolytic titration failed to reveal alternative pathway activation by the majority of strains of this group. Therapy aimed at supplying opsonins for these organisms will require the presence of type-specific antibody.  (+info)

Identification of a peptide from mammal albumins responsible for enhanced pigment production by group B streptococci. (7/1478)

The peptide from peptones responsible for enhanced pigment production by Streptococcus agalactiae in culture media has been isolated from a peptic digest of human albumin and has been identified as Ile-Ala-Arg-Arg-His-Pro-Tyr-Phe. The related heptapeptide lacking the N-terminal Ile also had pigment-enhancing activity. A sequence similarity search showed that these sequences are present only in mammal albumins.  (+info)

Alpha C protein as a carrier for type III capsular polysaccharide and as a protective protein in group B streptococcal vaccines. (8/1478)

The alpha C protein, a protective surface protein of group B streptococci (GBS), is present in most non-type III GBS strains. Conjugate vaccines composed of the alpha C protein and type III capsular polysaccharide (CPS) might be protective against most GBS infections. In this study, the type III CPS was covalently coupled to full-length, nine-repeat alpha C protein (resulting in III-alpha9r conjugate vaccine) or to two-repeat alpha C protein (resulting in III-alpha2r conjugate vaccine) by reductive amination. Initial experiments with the III-alpha9r vaccine showed that it was poorly immunogenic in mice with respect to both vaccine antigens and was suboptimally efficacious in providing protection in mice against challenge with GBS. Therefore, modified vaccination protocols were used with the III-alpha2r vaccine. Female mice were immunized three times with 0.5, 5, or 20 microgram of the III-alpha2r vaccine with an aluminum hydroxide adjuvant and bred. Ninety-five percent of neonatal mice born to dams immunized with the III-alpha2r vaccine survived challenge with GBS expressing type III CPS, and 60% survived challenge with GBS expressing wild-type (nine-repeat) alpha C protein; 18 and 17%, respectively, of mice in the negative control groups survived (P, <0.0001). These protection levels did not differ significantly from those obtained with the type III CPS-tetanus toxoid conjugate vaccine and the unconjugated two-repeat alpha C protein, which protected 98 and 58% of neonates from infection with GBS expressing type III CPS or the alpha C protein, respectively. Thus, the two-repeat alpha C protein in the vaccine was immunogenic and simultaneously enhanced the immunogenicity of type III CPS. III-alpha vaccines may be alternatives to GBS polysaccharide-tetanus toxoid vaccines, eliciting additional antibodies protective against GBS infection.  (+info)

A Comparative Investigation of Streptococcus Agalactiae Isolates from Fish and CattleA Comparative Investigation of Streptococcus Agalactiae Isolates from Fish and Cattle
Streptococcus agalactiae is the causative bacterium of streptococcosis and causes severe economic losses in wild and cultured fish and cattle, worldwide. In fish, infection can result in septicemia with hemorrhages on the body surface and in the external and internal organs. Streptococcus agalactiae may be isolated from brain, nares, head kidney and eye of infected fish. Streptococcus agalactiae also causes bovine mastitis and the organism can be isolated from milk samples. Streptococcus agalactiae is classified as Lancefields group B Streptococcus (GBS). There are also nine GBS capsular polysaccharide serotypes. Despite the significance of the disease, only limited information is available on the identification and characterization of the S. agalactiae isolates from fish and cattle. In the present study, GBS isolates from fish (n = 36), bottlenose dolphin (n=1) and cattle GBS isolates (n=10) were found to have a number of common phenotypic characteristics of the S. agalactiae reference strains ...
Group B streptococcus (GBS) and neonatal infections: the case for intrapartum chemoprophylaxis. - Semantic ScholarGroup B streptococcus (GBS) and neonatal infections: the case for intrapartum chemoprophylaxis. - Semantic Scholar
At the Royal Womens Hospital, Melbourne over an 8-year period (1981-1988) all public antenatal patients were screened at 32 weeks gestation for group B streptococcus (GBS). In a total of 30,197 livebirths there were no early onset neonatal GBS infections in infants of treated asymptomatic carrier mothers. By contrast there were 27 infections with 8 deaths in an unscreened control group of private patients (total livebirths 26,915). It is recommended that GBS screening occur antenatally at 28 weeks and that intrapartum chemoprophylaxis be offered at least to those carriers with obstetric risk factors.
Membrane Sweeping in Group B Streptococcus (GBS) Positive Patients - Full Text View - ClinicalTrials.govMembrane Sweeping in Group B Streptococcus (GBS) Positive Patients - Full Text View - ClinicalTrials.gov
Sweeping or stripping of the membranes (in this paper referred to as membrane sweeping) is a widely utilized technique to hasten delivery for women at 37+ weeks gestation. The process of membrane sweeping probably causes release of prostaglandins from the decidua and the cervix. The efficacy of membrane sweeping is well studied, and has been shown to increase the number of patients in labor within 72 hours, reduce the frequency of pregnancy continuing beyond 41 or 42 weeks, and reduce the frequency of formal induction of labor. Thus, it is a safe and practical option for women who wish to avoid inductions of labor or postterm pregnancies.. Group B streptococcus (GBS; streptococcus agalactiae) is a gram positive coccus that frequently colonizes the human genital tract. In pregnant women, GBS can cause urinary tract infections, chorioamnionitis, and postpartum endometritis. Newborn infants can also acquire GBS infection during passage through the vagina. Early onset GBS disease in the newborn ...
Human Streptococcus agalactiae strains in aquatic mammals and fish | BMC Microbiology | Full TextHuman Streptococcus agalactiae strains in aquatic mammals and fish | BMC Microbiology | Full Text
In humans, Streptococcus agalactiae or group B streptococcus (GBS) is a frequent coloniser of the rectovaginal tract, a major cause of neonatal infectious disease and an emerging cause of disease in non-pregnant adults. In addition, Streptococcus agalactiae causes invasive disease in fish, compromising food security and posing a zoonotic hazard. We studied the molecular epidemiology of S. agalactiae in fish and other aquatic species to assess potential for pathogen transmission between aquatic species and humans. Isolates from fish (n = 26), seals (n = 6), a dolphin and a frog were characterized by pulsed-field gel electrophoresis, multilocus sequence typing and standardized 3-set genotyping, i.e. molecular serotyping and profiling of surface protein genes and mobile genetic elements. Four subpopulations of S. agalactiae were identified among aquatic isolates. Sequence type (ST) 283 serotype III-4 and its novel single locus variant ST491 were detected in fish from Southeast Asia and shared a 3-set
Microbiology Society Journals | Molecular characteristics of Streptococcus agalactiae strains deficient in alpha-like protein...Microbiology Society Journals | Molecular characteristics of Streptococcus agalactiae strains deficient in alpha-like protein...
Streptococcus agalactiae (group B streptococci, GBS) are important human and animal pathogens, which can be subdivided based on different capsular polysaccharides and surface-anchored alpha-like proteins (Alps), as well as other proteins. Nearly all GBS strains possess an Alp (Alp GBS), although Alp-negative GBS (non-Alp GBS) do occur. In this study, 10 (1.1 %) of 932 clinical human GBS tested lacked an Alp encoding gene. All 10 strains were from patients with bloodstream infection, confirming that non-Alp GBS can be highly virulent. All non-Alp GBS expressed one or more of the surface-anchored proteins R3, Z1 and Z2, while less than 10 % of unselected clinical strains express any of these proteins. In contrast to Alp GBS, all non-Alp strains tested were PCR negative for the upstream sequence of the insertion site of the Alp encoding gene of Alp GBS. Genome sequencing showed that all but one of the 10 clinical non-Alp strains and the non-Alp reference strain CNCTC 10/84 lacked a region surrounding the
Epidemiology of invasive group B streptococcal disease in infants from urban area of South China, 2011-2014Epidemiology of invasive group B streptococcal disease in infants from urban area of South China, 2011-2014
Background: Group B Streptococcus (GBS) is a leading cause of morbidity and mortality in infants in both developed and developing countries. To our knowledge, only a few studies have been reported the clinical features, treatment and outcomes of the GBS disease in China. The severity of neonatal GBS disease in China remains unclear. Population-based surveillance in China is therefore required. Methods: We retrospectively collected data of ,3 months old infants with culture-positive GBS in sterile samples from three large urban tertiary hospitals in South China from Jan 2011 to Dec 2014. The GBS isolates and their antibiotic susceptibility were routinely identified in clinical laboratories in participating hospitals. Serotyping and multi-locus sequence typing (MLST) were also conducted for further analysis of the neonatal GBS disease. Results: Total 70 cases of culture-confirmed invasive GBS infection were identified from 127,206 live births born in studying hospitals, giving an overall incidence ...
Awareness of Primary Care Physicians about Pregnancy Screening of Group B Streptococcus Infection at Prince Sultan Military...Awareness of Primary Care Physicians about Pregnancy Screening of Group B Streptococcus Infection at Prince Sultan Military...
Awareness of Primary Care Physicians about Pregnancy Screening of Group B Streptococcus Infection at Prince Sultan Military Medical City, Riyadh, Saudi Arabia
Risk Factors for Early-onset Group B Streptococcal Sepsis: Estimation of Odds Ratios by Critical Literature Review | American...Risk Factors for Early-onset Group B Streptococcal Sepsis: Estimation of Odds Ratios by Critical Literature Review | American...
Early-onset group B streptococcal sepsis (EOGBS) has been the leading cause of death attributable to infection in newborn infants for nearly 3 decades,1 with ,6000 cases a year in the United States.2 The attack rate has not changed over the past 20 years, but the case-fatality rate has declined from ∼50% to between 10% and 15%.3-5 Long-term morbidity among survivors, particularly neurodevelopmental disabilities in those with meningitis, remain distressingly common.6-9 EOGBS disease may be rapidly progressive and many infants (especially at term) do not exhibit clinical signs of infection initially5; therefore, empirical therapy is often initiated because of clinical risk or minimal signs of disease and continued until infection has been excluded by laboratory studies and a period of observation. This aggressive approach has been associated with improved outcomes for infants with this disease,10 ,11 but also may lead to acute-care hospitalization and treatment of ,100 000 newborn infants yearly ...
Intrapartum antibiotic prophylaxis for Group B Streptococcus and risks of unnecessary antibiotics | Archivio della ricerca dell...Intrapartum antibiotic prophylaxis for Group B Streptococcus and risks of unnecessary antibiotics | Archivio della ricerca dell...
We much appreciated the Clinical Opinion by Turrentine1 regarding intrapartum antibiotic prophylaxis (IAP) for the prevention of neonatal group B Streptococcus (GBS) early-onset sepsis. The author raises concerns about cases of inadequate duration (,4 hours) of IAP before delivery. Indeed, as much as 25-40% of GBS-colonized women will not receive the 4 hours of IAP that are recommended by guidelines. To optimize fetal exposure, Turrentine recommends strategies such as (1) to postpone artificial rupture of membranes (or administration of oxytocin) until 4 hours IAP is completed and (2) to start IAP before the initiation of uterotonic agents in women who are admitted for induction of labor. Although the first step is feasible, a woman with a Bishop score ,5 may be exposed to several antibiotic doses until delivery. The minimum duration of IAP for the prevention of early-onset GBS sepsis remains uncertain, because existing data are conflicting. There is evidence that bactericidal levels of ...
Invasive neonatal Group B streptococcal infection, over a 10 year period, in West Hertfordshire, UK. | AbstractInvasive neonatal Group B streptococcal infection, over a 10 year period, in West Hertfordshire, UK. | Abstract
Introduction: Group B Streptococcus (GBS) infection is the most important cause of neonatal infection in developed and developing countries. UK Nation..
A unique serine-rich repeat protein (Srr-2) and novel surface antigen (ε) associated with a virulent lineage of serotype III...A unique serine-rich repeat protein (Srr-2) and novel surface antigen (ε) associated with a virulent lineage of serotype III...
Group B streptococci (GBS) are pathogens of both neonates and adults, with serotype III strains in particular being associated with invasive disease and meningitis. In this study, a novel GBS surface antigen, ε, was found to be co-expressed with the previously reported δ antigen on an identical subset of serotype III GBS. Expression of δ/ε on the surface of serotype III GBS was shown to distinguish the restriction digest pattern (RDP) III-3 and multilocus sequence typing (ST)-17 lineage. ε-Specific antibodies were reactive with a unique, high-molecular-mass, serine-rich repeat protein (Srr-2) found exclusively in RDP III-3 strains. The gene encoding Srr-2 was located within a putative accessory secretory locus that included secY2 and secA2 homologues and had a genetic organization similar to that of the secY2/A2 locus of staphylococci. In contrast, serotype III δ/ε-negative strains and strains representative of serotypes Ia, Ib, Ic and II shared a common Srr-encoding gene, srr-1, and an
The alpha C protein mediates internalization of group B Streptococcus within human cervical epithelial cells<...The alpha C protein mediates internalization of group B Streptococcus within human cervical epithelial cells<...
TY - JOUR. T1 - The alpha C protein mediates internalization of group B Streptococcus within human cervical epithelial cells. AU - Bolduc, G. R.. AU - Baron, M. J.. AU - Gravekamp, C.. AU - Lachenauer, C. S.. AU - Madoff, L. C.. PY - 2002/11/1. Y1 - 2002/11/1. N2 - Group B Streptococcus (GBS) is the leading cause of bacterial chorioamnionitis and neonatal pneumonia, sepsis, and meningitis. Deletion of the alpha C protein gene (bca) attenuates the virulence of GBS in an animal model; significant survival differences in the first 24 h of infection suggest a pathogenic role for the alpha C protein early in the infection process. We examined the role of alpha C protein in the association between GBS and mucosal surfaces using a human cervical epithelial cell line, ME180. Fluorescent and confocal microscopy and flow cytometry demonstrated that 9-repeat alpha C protein binds to the surface of ME180 cells. Isolated N-terminal region of this protein also binds to these cells and competitively inhibits ...
Antibiotic Susceptibilities and Serotyping of Clinical Streptococcus Agalactiae Isolates | AVESİSAntibiotic Susceptibilities and Serotyping of Clinical Streptococcus Agalactiae Isolates | AVESİS
Objective: Streptococcus agalactiae (Group B streptococci, GBS) are frequently responsible for sepsis and meningitis seen in the early weeks of life. GBS may cause perinatal infection and premature birth in pregnant women. The aim of this study was to serotype GBS strains isolated from clinical samples and evaluate their serotype distribution according to their susceptibilities to antibiotics and isolation sites. ...
Group B Streptococcal SepsisGroup B Streptococcal Sepsis
This page includes the following topics and synonyms: Group B Streptococcal Sepsis, Group B Streptococcus, GBS Sepsis, Neonatal Sepsis from GBS, Group B Streptococcal Pneumonia.
SID.ir | ESTIMATION OF GROUP B STREPTOCOCCUS COLONIZATION IN HIGH-RISK NEONATES BY PCR AND STANDARD CULTURESID.ir | ESTIMATION OF GROUP B STREPTOCOCCUS COLONIZATION IN HIGH-RISK NEONATES BY PCR AND STANDARD CULTURE
Download Free Full-Text of an article ESTIMATION OF GROUP B STREPTOCOCCUS COLONIZATION IN HIGH-RISK NEONATES BY PCR AND STANDARD CULTURE
Trends in Perinatal Group B Streptococcal Disease - United States, 2000-2006Trends in Perinatal Group B Streptococcal Disease - United States, 2000-2006
Group B Streptococcus (GBS) is a leading infectious cause of neonatal morbidity and mortality in the United States. The bacterium, a common colonizer of the maternal genital tract, can infect the fetus during gestation, causing fetal death. GBS also can be acquired by the fetus during passage throug …
Multilocus Sequence Typing of Swedish Invasive Group B Streptococcus Isolates Indicates a Neonatally Associated Genetic Lineage...Multilocus Sequence Typing of Swedish Invasive Group B Streptococcus Isolates Indicates a Neonatally Associated Genetic Lineage...
The group B streptococcus is a common cause of neonatal diseases, such as pneumonia, septicemia, and meningitis; but GBS is also known to cause invasive as well as noninvasive infections in adults. However, little is known about the genetic relationship among the isolates, the diversity of virulence, and possible differences in tropism among GBS isolates. We used MLST analysis and screened for the presence of mobile genetic elements among clinical isolates collected in two geographic regions in Sweden during a 10-year time span to investigate the population structure and dynamics among invasive GBS isolates.. By MLST analysis, 158 GBS invasive isolates collected from northern and western parts of Sweden were resolved into 29 STs and grouped into six genetic lineages, including five major clonal complexes and one singleton. The highly clonal structure has also been shown by a number of other methods, such as pulsed-field gel electrophoresis, multilocus enzyme electrophoresis typing, and randomly ...
Trend of isolation and serotypes of group B streptococci in Korea.Trend of isolation and serotypes of group B streptococci in Korea.
Group B streptococci (GBS) neonatal infection, a prevalent disease in western countries, is considered rare in Korea. GBS neonatal infection is known to be often due to serotype III organisms, but the serotypes in Korea have not been reported. In this study, GBS were frequently isolated from specimens of genitalia, urine and various pus. Among the 186 isolates 14 (7.5%) were from neonates, two with concomitant bacteremia and meningitis and one with pneumonia. Frequently isolated GBS serotypes were Ib (9.2%), Ib/c (26.6%) and III/R (23.9%). Change of frequently isolated serotypes during the study was noted, but JM9 which became increasingly isolated in Japan was not found. It is concluded that less prevalence of severe neonatal GBS infection in Korea is not due to the absence of serotype III, but possibly due to low genital carriage rate of GBS by pregnant women.
The enhancement of biofilm formation in Group B streptococcal isolates at vaginal pH<...The enhancement of biofilm formation in Group B streptococcal isolates at vaginal pH<...
TY - JOUR. T1 - The enhancement of biofilm formation in Group B streptococcal isolates at vaginal pH. AU - Ho, Yueh Ren. AU - Li, Chien Ming. AU - Yu, Chen Hsiang. AU - Lin, Yuh Jyh. AU - Wu, Ching Ming. AU - Harn, I. Chen. AU - Tang, Ming Jer. AU - Chen, Yi Ting. AU - Shen, Fang Chi. AU - Lu, Chien Yi. AU - Tsai, Tai Chun. AU - Wu, Jiunn Jong. PY - 2013/4/1. Y1 - 2013/4/1. N2 - Group B streptococcus (GBS) is a common asymptomatic colonizer in acidic vagina of pregnant women and can transmit to newborns, causing neonatal pneumonia and meningitis. Biofilm formation is often associated with bacterial colonization and pathogenesis. Little is known about GBS biofilm and the effect of environmental stimuli on their growth along with biofilm formation. The objective of this study was to investigate the survival and biofilm formation of GBS, isolated from pregnant women, in nutrient-limited medium under various pH conditions. Growth and survival experiments were determined by optical density and viable ...
Streptococcus agalactiae Lehmann and Neumann ATCC ® BAA-611D-5&traStreptococcus agalactiae Lehmann and Neumann ATCC ® BAA-611D-5&tra
Streptococcus agalactiae ATCC ® BAA-611D-5™ Designation: Genomic DNA from Streptococcus agalactiae strain 2603 V/R TypeStrain=False Application:
Chemoprophylaxis of early-onset group B streptococcal disease in 1999.Chemoprophylaxis of early-onset group B streptococcal disease in 1999.
The evolution of the guidelines for selective intrapartum chemoprophylaxis (SIC) of group B streptococcal early-onset disease is reviewed here. To assess the benefits of the risk-based and culture-based strategies for prevention, observational studie
Hyperinvasive neonatal group B streptococcus has arisen from a bovine ancestor. - Nuffield Department of MedicineHyperinvasive neonatal group B streptococcus has arisen from a bovine ancestor. - Nuffield Department of Medicine
The genetic relatedness and evolutionary relationships between group B streptococcus (GBS) isolates from humans and those from bovines were investigated by phylogenetic analysis of multilocus sequence typing data. The collection of isolates consisted of 111 GBS isolates from cows with mastitis and a diverse global collection of GBS isolates from patients with invasive disease (n = 83) and carriers (n = 69). Cluster analysis showed that the majority of the bovine isolates (93%) grouped into one phylogenetic cluster. The human isolates showed greater diversity and clustered separately from the bovine population. However, the homogeneous human sequence type 17 (ST-17) complex, known to be significantly associated with invasive neonatal disease, was the only human lineage found to be clustered within the bovine population and was distinct from all the other human lineages. Split decomposition analysis revealed that the human isolate ST-17 complex, the major hyperinvasive neonatal clone, has recently arisen
The Streptococcus agalactiae stringent response enhances virulence and persistence in human bloodThe Streptococcus agalactiae stringent response enhances virulence and persistence in human blood
Streptococcus agalactiae (group B Streptococcus [GBS]) causes serious infections in neonates. We previously reported a transposon sequencing (Tn-seq) system for performing genomewide assessment of gene fitness in GBS. In order to identify molecular mechanisms required for GBS to transition from a mucosal commensal lifestyle to bloodstream invasion, we performed Tn-seq on GBS strain A909 with human whole blood. Our analysis identified 16 genes conditionally essential for GBS survival in blood, of which 75% were members of the capsular polysaccharide (cps) operon. Among the non-cps genes identified as conditionally essential was relA, which encodes an enzyme whose activity is central to the bacterial stringent response-a conserved adaptation to environmental stress. We used blood coincubation studies of targeted knockout strains to confirm the expected growth defects of GBS deficient in capsule or stringent response activation. Unexpectedly, we found that the relA knockout strains demonstrated ...
Adoption of Perinatal Group B Streptococcal Disease Prevention Recommendations by Prenatal-Care Providers --- Connecticut and...Adoption of Perinatal Group B Streptococcal Disease Prevention Recommendations by Prenatal-Care Providers --- Connecticut and...
Group B streptococcal (GBS) infections are the leading bacterial cause of serious neonatal disease in the United States (1). In 1996, in collaboration with the American Academy of Pediatrics and the American College of Obstetricians and Gynecologists, CDC issued consensus guidelines for preventing perinatal GBS disease (2--4). These guidelines recommend using either a screening-based or a risk-based strategy to identify women who should receive intrapartum antimicrobial prophylaxis. To assess adoption of the GBS disease prevention guidelines, the Connecticut and Minnesota state health departments surveyed prenatal-care providers during January--April 1998. This report presents the survey findings, which indicate that most prenatal-care providers in Connecticut and Minnesota have adopted perinatal GBS disease prevention policies and that strategy choice may vary by state and provider type. In Connecticut, surveys were mailed to all (n=576) licensed obstetricians/gynecologists (OBs). Group ...
Group B Streptococcal Disease | Reston Hospital CenterGroup B Streptococcal Disease | Reston Hospital Center
Learn more about Group B Streptococcal Disease at Reston Hospital Center DefinitionCausesRisk FactorsSymptomsDiagnosisTreatmentPreventionrevision ...
Typing of Streptococcus agalactiae (Lancefield group B) | SpringerLinkTyping of Streptococcus agalactiae (Lancefield group B) | SpringerLink
A two-stage typing scheme in routine use in this laboratory is described. The strains of group B streptococci (GBS) are first serotyped and then, if necessary, phage-typing is performed. Serotyping...
Genetic and pathogenic difference between Streptococcus agalactiae serotype Ia fish and human isolates | BMC Microbiology |...Genetic and pathogenic difference between Streptococcus agalactiae serotype Ia fish and human isolates | BMC Microbiology |...
Streptococcus are pathogenic to cause streptococcal disease for humans and animals. Among Streptococcus spp., Gram-positive Streptococcus agalactiae (group B streptococcus; GBS) is a normal human gastrointestinal and genitourinary flora. Therefore, GBS infect more commonly the vaginas of women, especially more prevalent in the pregnant woman than those of non-pregnant woman [1] and causes early-onset or late-onset sepsis and meningitis in newborns. In fish, the major pathogenic species that cause streptococcosis are S. agalactiae (GBS), S. dysgalactiae, S. iniaee and Lactococcus garvieae, which also infect human. Multiplex PCR has been developed to differentiate these species and to identify serotypes [2], especially the GBS serotypes [3, 4].. Streptococcosis is an important disease in fish. After infection, fishes may suffer meningitis and septicemia in common [5]; however, other syndromes may be associated with fish species [6]. Streptococcal infection has been reported in rainbow trout in ...
Frontiers | Increased Risk of Group B Streptococcus Invasive Infection in HIV-Exposed but Uninfected Infants: A Review of the...Frontiers | Increased Risk of Group B Streptococcus Invasive Infection in HIV-Exposed but Uninfected Infants: A Review of the...
Group B streptococcus (GBS) is a major cause of neonatal sepsis and mortality worldwide. Studies from both developed and developing countries have shown that HIV exposed but uninfected (HEU) infants are at increased risk of infectious morbidity, as compared to HIV unexposed uninfected infants (HUU). A higher susceptibility to GBS infections has been reported in HEU infants, particularly late-onset diseases (LOD) and more severe manifestations of GBS diseases. We review here the possible explanations for increased susceptibility to GBS infection. Maternal GBS colonization during pregnancy is a major risk factor for early-onset GBS invasive disease but colonization rates are not higher in HIV-infected compared to HIV-uninfected pregnant women, while selective colonization with more virulent strains in HIV-infected women is suggested in some studies. Lower serotype specific GBS maternal antibody transfer and quantitative and qualitative defects of innate immune responses in HEU infants may play a role in
Attitudes towards vaccination against group B streptococcus in pregnancy | Archives of Disease in ChildhoodAttitudes towards vaccination against group B streptococcus in pregnancy | Archives of Disease in Childhood
Group B streptococcus (GBS) is the commonest cause of sepsis and meningitis in the neonatal period in the UK with case numbers similar to that of meningococcal disease in older children.1 Mortality is around 10%, and 50% of GBS meningitis survivors are left with long-term neurodevelopmental sequelae.1 The selective use of intravenous antibiotics during labour has been shown to reduce the incidence of early-onset GBS infection in neonates, but has no effect on late-onset disease.2 Therefore, a better method of protecting infants is required ...
Group B Streptococcus Infection in NewbornsGroup B Streptococcus Infection in Newborns
Group B streptococcus (strep) is a type of bacteria. It can be found in the digestive tract, urinary tract, and genital area of adults. About 1 in 4 pregnant women carry GBS in their rectum or vagina. During pregnancy, the mother can pass the infection to the baby. The fetus can get GBS during pregnancy. Newborns can get it from the mothers genital tract during delivery.
Group B Streptococcus Infection in BabiesGroup B Streptococcus Infection in Babies
Group B streptococcus (strep) is a type of bacteria. It can be found in the digestive tract, urinary tract, and genital area of adults. About 1 in 4 pregnant women carry GBS in their rectum or vagina. During pregnancy, the mother can pass the infection to the baby. The fetus can get GBS during pregnancy. Newborns can get it from the mothers genital tract during delivery.
Progress toward a group B streptococcal vaccine<...Progress toward a group B streptococcal vaccine<...
TY - JOUR. T1 - Progress toward a group B streptococcal vaccine. AU - Song, Joon-Young. AU - Lim, Jae Hyang. AU - Lim, Sangyong. AU - Yong, Zhi. AU - Seo, Ho Seong. PY - 2018/11/2. Y1 - 2018/11/2. N2 - Streptococcus agalactiae (group B Streptococcus, GBS) is a leading cause of severe invasive disease in neonate, elderly, and immunocompromised patients worldwide. Despite recent advances in the diagnosis and intrapartum antibiotic prophylaxis (IAP) of GBS infections, it remains one of the most common causes of neonatal morbidity and mortality, causing serious infections. Furthermore, recent studies reported an increasing number of GBS infections in pregnant women and elderly. Although IAP is effective, it has several limitations, including increasing antimicrobial resistance and late GBS infection after negative antenatal screening. Maternal immunization is the most promising and effective countermeasure against GBS infection in neonates. However, no vaccine is available to date, but two types of ...
Prevention and Management of Infants With Suspected or Proven Neonatal Sepsis | American Academy of PediatricsPrevention and Management of Infants With Suspected or Proven Neonatal Sepsis | American Academy of Pediatrics
In 2010, the Centers for Disease Control and Prevention in collaboration with the American Academy of Family Physicians, American Academy of Pediatrics (AAP), American College of Nurse-Midwives, American College of Obstetricians and Gynecologists, and American Society for Microbiology published revised group B streptococcal (GBS) guidelines entitled "Prevention of perinatal group B streptococcal disease: revised guidelines from CDC 2010" in the Morbidity and Mortality Weekly Report (MMWR).1 The recommendations were endorsed by all the collaborating organizations including AAP after review by the Committee on Infectious Diseases (COID) and the Committee on Fetus and Newborn (COFN) and after AAP Board approval. In the report, a revised algorithm for "Secondary prevention of early-onset group B streptococcal disease" was included. This algorithm represented the expert opinions of the technical working group (based on current literature available at that time). In 2011, the COID and the COFN ...
Streptococcus agalactiaeStreptococcus agalactiae
... is a beta-hemolytic gram-positive bacteria. It is also known as "Group B streptococcus" (or GBS). They are normally found in the gastroinstesinal flora in humans and in the female urogenital tract and rectum. S. agalatiae can act in a variety of manners, depending on the condition of the body it is residing in. It is a diplococcal gram-positive, non acid-fast bacterium found in pairs or chains. It is inmotile, does not form spores, contains the group B Lancefield antigen, and catalase-free, where it does not catalyze the reduction of hydrogen peroxide.. S. agalactiae is very sensitive to bile and will lyse if it is in contact with it as a result. The detection of S. agalactiae can be achieved in various ways, such as a CAMP (Christie Atkins Munch-Petersen) test, can be performed; however if its presence is need of controlling, i.e. infection, it can be very difficult due to the fact that it is naturally occuring in the human body. S. agalactiae is capable of serious ...
Type-specific protection of neonatal rats from lethal group B streptococcal infection by immune sera obtained from human...Type-specific protection of neonatal rats from lethal group B streptococcal infection by immune sera obtained from human...
Sera obtained from human volunteers at 6 weeks after vaccination with highly purified type III polysaccharide antigen prepared from a group B Streptococcus, strain M732, were found to protect neonatal rats from otherwise lethal infection by the homologous strain. The specific antibody content of the sera, expressed in micrograms of antibody protein per milliliter, was determined by an enzyme-linked immunosorbent assay in conjunction with quantitative precipitin analysis. For two sera studied in detail, the protective dose of antibody for 50% of the animals was 0.4 micrograms. Immune serum obtained from a volunteer who received type II polysaccharide vaccine was not protective against type III infection. Absorption of anti-type III serum by quantitative precipitation of antibodies with type III polysaccharide completely removed the passive protective activity of the serum. The results show that antibodies induced in humans by purified type II polysaccharide give serotype-specific protection in an ...
NewYork-Presbyterian Queens - High-Risk Pregnancy - Group B StreptococcusNewYork-Presbyterian Queens - High-Risk Pregnancy - Group B Streptococcus
About 25 percent of pregnant women carry GBS in their rectum or vagina. In the pregnant mother, GBS infection may cause chorioamnionitis (a severe infection of the placental tissues) and postpartum (after birth) infection. Urinary tract infections caused by GBS can lead to preterm labor and birth.. Newborns can contract GBS during pregnancy, or from the mothers genital tract during labor and delivery. GBS is the most common cause of life-threatening infections in newborns, including pneumonia and meningitis. If a pregnant woman who is a group B strep carrier does not get antibiotics at the time of delivery, her baby has a 1 in 200 chance of developing GBS disease. Nearly 75 percent of the cases of GBS disease among newborns occur in the first week of life, called early-onset disease. Premature babies are more susceptible to GBS infection than full-term babies.. Although it is very rare, GBS infection may also develop in babies one week to several months after birth, called late-onset disease. ...
NewYork-Presbyterian Queens - Group B StreptococcusNewYork-Presbyterian Queens - Group B Streptococcus
About 25 percent of pregnant women carry GBS in their rectum or vagina. In the pregnant mother, GBS infection may cause chorioamnionitis (a severe infection of the placental tissues) and postpartum (after birth) infection. Urinary tract infections caused by GBS can lead to preterm labor and birth.. Newborns can contract GBS during pregnancy, or from the mothers genital tract during labor and delivery. GBS is the most common cause of life-threatening infections in newborns, including pneumonia and meningitis. If a pregnant woman who is a group B strep carrier does not get antibiotics at the time of delivery, her baby has a 1 in 200 chance of developing GBS disease. Nearly 75 percent of the cases of GBS disease among newborns occur in the first week of life, called early-onset disease. Premature babies are more susceptible to GBS infection than full-term babies.. Although it is very rare, GBS infection may also develop in babies one week to several months after birth, called late-onset disease. ...
Correlation of maternal antibody deficiency with susceptibility to neonatal group B streptococcal infection. - PubMed - NCBICorrelation of maternal antibody deficiency with susceptibility to neonatal group B streptococcal infection. - PubMed - NCBI
PubMed comprises more than 30 million citations for biomedical literature from MEDLINE, life science journals, and online books. Citations may include links to full-text content from PubMed Central and publisher web sites.
Effects of complement regulators bound to Escherichia coli K1 and Group B Streptococcus on the interaction with host cells.Effects of complement regulators bound to Escherichia coli K1 and Group B Streptococcus on the interaction with host cells.
Escherichia coli K1 and Group B Streptococcus (GBS) are the most common bacteria that cause meningitis during the neonatal period. Complement, the first line of defence in the host, acts on these bacteria to opsonize with various components of comple
Enhanced Expression of lmb Gene Encoding Laminin-Binding Protein in Streptococcus agalactiae Strains Harboring IS1548 in scpB...Enhanced Expression of lmb Gene Encoding Laminin-Binding Protein in Streptococcus agalactiae Strains Harboring IS1548 in scpB...
PubMed Central Canada (PMC Canada) provides free access to a stable and permanent online digital archive of full-text, peer-reviewed health and life sciences research publications. It builds on PubMed Central (PMC), the U.S. National Institutes of Health (NIH) free digital archive of biomedical and life sciences journal literature and is a member of the broader PMC International (PMCI) network of e-repositories.
Anti Streptococcus agalactiae Antibody, clone 224/46 | Bio-RadAnti Streptococcus agalactiae Antibody, clone 224/46 | Bio-Rad
|strong|Mouse anti |em|Streptococcus agalactiae|/em| antibody, clone 224/46|/strong| recognizes |em|Streptococcus agalactiae|/em| a gram-positive streptococcus characterized by the presence of the gro…
Detection and quantification of Streptococcus agalactiae from aquaculture with molecular means - Enlighten: ThesesDetection and quantification of Streptococcus agalactiae from aquaculture with molecular means - Enlighten: Theses
The aim of this research was to improve the detection and diagnosis of Streptococcus agalactiae in aquaculture, as well as developing methods to improve the study of this bacteria in the laboratory. Difficulties in specifically detecting the presence of S. agalactiae from samples of an aquatic nature had been observed using primers developed for detecting S. agalactiae in other hosts. Cross reaction of the primers with closely related fish pathogens, or environmental bacteria had been observed when using these primers to identify aquaculture derived samples. A rapid, sensitive and specific Polymerase Chain Reaction (PCR) assay based on the S. agalactiae groEL gene was designed in order to quickly diagnose S. agalactiae from an infected or dead fish. It is important for rapid diagnosis of this disease as mortalities among the fish population can spread extremely rapidly. In the time taken to culture bacteria from a diseased fish in the lab, high levels of mortality among infected fish may already ...
Rapid Diagnostic Test for Detection of Group B Streptococcus in Pregnancy - Full Text View - ClinicalTrials.govRapid Diagnostic Test for Detection of Group B Streptococcus in Pregnancy - Full Text View - ClinicalTrials.gov
Intrapartum infection with group B streptococci (GBS) may lead to untoward neonatal sequelae such as pneumonia, septicemia, and meningitis. Several clinical trials have demonstrated that the use of intrapartum intravenous antibiotic prophylaxis (with penicillin being the agent of choice due to a narrower spectrum of antimicrobial activity) is highly effective to prevent early-onset neonatal GBS infections. It has been estimated that 10% to 30% of all pregnant women are GBS carriers, and it is likely that GBS colonizes virtually every female at some point. Consensus guidelines from the Centers for Disease Control and Prevention (CDC), American College of Obstetricians and Gynecologists (ACOG), and the American Academy of Pediatrics, recommend prenatal screening at 35 to 37 weeks of gestation and chemoprophylaxis at delivery based upon this culture result. The CDC-recommended method for detecting GBS colonization involves collecting a combined vaginal and rectal specimen and culturing the organism ...
Evaluation of rapid methods of identifying group B streptococci. | Journal of Clinical PathologyEvaluation of rapid methods of identifying group B streptococci. | Journal of Clinical Pathology
Rapid methods of identifying Lancefield group B streptococci were compared to the standard Fullers extraction method. Such tests as sodium hippurate hydrolysis, bile tolerance, aesculin hydrolysis, pyruvate fermentation, Camp factor, pigmentation, and bacitracin haemolysis were tested on both routine clinical specimens and National Collection of Type Culture strains. The results show that pigmentation on Islamss medium was the most definitive test available rapidly to identify group B streptococci.. ...
Identification and molecular characterization of a S. agalactiae strain lacking the capsular locusIdentification and molecular characterization of a S. agalactiae strain lacking the capsular locus
During a national surveillance program on Group B streptococci (GBS) maternal carriage and neonatal infections, a GBS strain isolated from a pregnant womans vagino-rectal swab was non typable by either serological or molecular methods. Further molecular characterization demonstrated that the strain …
Molecular Characterization of Group B Streptococcus Serotypes By Multiplex Polymerase Chain ReactionMolecular Characterization of Group B Streptococcus Serotypes By Multiplex Polymerase Chain Reaction
REFERENCES. 1 Stoll BJ, Hansen NI, Sánchez PJ, Faix RG, Poindexter BB, Van Meurs KP, et al. Early onset neonatal sepsis: the burden of group B streptococcal and E. coli disease continues. Pediatrics. 2011; 127(5):817-26. DOI:10.1542/peds.2010-2217 [ Links ] 2 Fiolo K, Zanardi CE, Salvadego M, Bertuzzo CS, Amaral E, Calil R, et al. Taxa de infecção e sorotipos de Streptococcus agalactiae em amostras de recém-nascidos infectados na cidade de Campinas (SP), Brasil. Rev Bras Ginecol Obstet. 2012; 34(12):544-9. DOI:10.1590/S0100-72032012001200003. [ Links ] 3 Dutra VG, Alves VM, Olendzki AN, Dias CA, de Bastos AF, Santos GO, et al. Streptococcus agalactiae in Brazil: serotype distribution, virulence determinants and antimicrobial susceptibility. BMC Infect Dis. 2014;14:323. DOI:10.1186/1471-2334-14-323. [ Links ] 4 Ko DW, Zurynski Y, Gilbert GL. GBS Study Group. Group B streptococcal disease and genotypes in Australian infants. J Paediatr Child Health. 2015;51(8): 808-14. DOI:10.1111/jpc.12830 [ ...
Streptococcus agalactiae | Pathway MedicineStreptococcus agalactiae | Pathway Medicine
S. agalactiae, more popularly known as "Group B Streptococcus (GBS)", primarily affects neonates who acquire the infection as they pass through the birth canal during delivery i.e. (Mother-to-Child Transmission). The organism is a part of the normal vaginal flora in a third of women. FYI: All women are now screened for GBS colonization in their rectovaginal mucosa during the third trimester and if positive are treated with antibiotics immediately prior to delivery, an intervention that has been shown to reduce rates of neonatal sepsis ...
Guidelines for Group B streptococcus | ADC Fetal & Neonatal EditionGuidelines for Group B streptococcus | ADC Fetal & Neonatal Edition
Editor-As highlighted in a recent edition ofFetal and Neonatal, there is increasing concern about the previously unreported high levels of neonatal group B Streptococcal (GBS) infections in the UK.1 2 It is important that we have statistics for regional variations in GBS infection so that we can to produce evidence based guidelines. It is also important that we are clear about the data on which we base our recommendations.. In the commentary which followed our recent paper,2 Nicoll and Heath1 refer to the incidence at which a risk factor based versus a screening approach to the prevention of Group B Streptococcus would be cost effective, quoting from a commentary by Isaacs.3 The original article by Mohle-Boetaniet al from which these data were derived,4 actually gives figures of , 0.65 and , 1.45/1000 live births at which a risk factor and screening based approach, respectively would be cost effective. This contrasts with those quoted by Isaacs, and Nicoll and Heath, of 0.6 and , 1.2/1000 live ...
Late-onset invasive group B streptococcal infection with serotype VIII in a neonate having congenital biliary atresia<...Late-onset invasive group B streptococcal infection with serotype VIII in a neonate having congenital biliary atresia<...
TY - JOUR. T1 - Late-onset invasive group B streptococcal infection with serotype VIII in a neonate having congenital biliary atresia. AU - Takei, Tomoaki. AU - Chiba, Naoko. AU - Fujita, Hisayo. AU - Morozumi, Miyuki. AU - Kuwata, Yusuke. AU - Kishii, Kozue. AU - Ubukata, Kimiko. AU - Iwata, Satoshi. AU - Takahashi, Takashi. PY - 2013/2. Y1 - 2013/2. N2 - A female newborn was admitted to our department 15 days after birth for insufficient sucking and jaundice. The patients blood and urine cultures were both positive for group B streptococcal (GBS) infection. A maternal vaginal sample at 35 weeks gestation was negative for GBS in culture-based microbiologic screening. The patient recovered shortly after receiving systemic antibiotic therapy. On the basis of clinical evidence of white stool and progressive jaundice, we suspected that the newborn had complications related to congenital biliary atresia (CBA); surgery was performed. Isolates from the mothers vaginal sample obtained when the ...
Purpura fulminans and late onset group B streptococcal sepsis in a premature twin<...Purpura fulminans and late onset group B streptococcal sepsis in a premature twin<...
TY - JOUR. T1 - Purpura fulminans and late onset group B streptococcal sepsis in a premature twin. AU - Elayappen, Avanikkha. AU - Jain, Sunil K.. AU - Loeffelholz, Michael J.. AU - Patel, Janak. PY - 2014/5/12. Y1 - 2014/5/12. N2 - Introduction: Purpura fulminans (PF) is a skin manifestation due to hemorrhagic infarction caused by intravascular thrombosis secondary to bacterial infections or deficiency of anticoagulants such as protein C and protein S. Neonatal PF is a rare but potentially disabling disorder associated with a high mortality and severe long term morbidity in those who survive. Case description: We report a case of a premature infant who developed extensive PF due to late onset group B streptococcus sepsis. Despite early identification and initiation of antibiotic therapy in our patient, PF progressed rapidly, leading to autoamputation of fingers and toes and severe brain injury. Conclusion: In conclusion, our case highlights the severe sequelae of PF due to late onset GBS sepsis ...
Group B streptococci | definition of group B streptococci by Medical dictionaryGroup B streptococci | definition of group B streptococci by Medical dictionary
Looking for online definition of group B streptococci in the Medical Dictionary? group B streptococci explanation free. What is group B streptococci? Meaning of group B streptococci medical term. What does group B streptococci mean?
Erratum: Complete genome sequence of Streptococcus agalactiae serotype III, multilocus sequence type 283 strain SG-M1 [Genome...Erratum: Complete genome sequence of Streptococcus agalactiae serotype III, multilocus sequence type 283 strain SG-M1 [Genome...
Mehershahi, K.S, Hsu, L.Y, Koh, T.H, Chen, S.L, on behalf of the Singapore Streptococcus agalactiae Working Group (2015). Erratum: Complete genome sequence of Streptococcus agalactiae serotype III, multilocus sequence type 283 strain SG-M1 [Genome Announc, 3, 5 (2015) (e01188-15)], DOI: 10.1128/genomeA.01188-15. Genome Announcements 3 (6) : e0158515. [email protected] Repository. https://doi.org/10.1128/genomeA.01585- ...
Pathogens | Free Full-Text | Evaluation of the Immunomodulatory Properties of Streptococcus suis and Group B Streptococcus...Pathogens | Free Full-Text | Evaluation of the Immunomodulatory Properties of Streptococcus suis and Group B Streptococcus...
Streptococcus suis and group B Streptococcus (GBS) are encapsulated streptococci causing septicemia and meningitis. Antibodies (Abs) against capsular polysaccharides (CPSs) have a crucial protective role, but the structure/composition of the CPS, including the presence of sialic acid, may interfere with the generation of anti-CPS Ab responses. We investigated the features of the CPS-specific Ab response directed against S. suis serotypes 2 and 14 and GBS serotypes III and V after infection or immunization with purified native or desialylated CPSs in mice. Whereas S. suis-infected mice developed a very low/undetectable CPS-specific IgM response, significant anti-CPS IgM titers were measured in GBS-infected animals (especially for type III GBS). No isotype switching was detected in S. suis- or GBS-infected mice. While the expression of sialic acid was essential for the immunogenicity of purified GBS type III CPS, this sugar was not responsible for the inability of purified S. suis types 2, 14 and GBS type
Mucin model for group B type III streptococcal infection in mice. | Infection and ImmunityMucin model for group B type III streptococcal infection in mice. | Infection and Immunity
An experimental murine infection was established by the intraperitoneal injection of a log-phase culture of a laboratory reference strain of Streptococcus agalactiae, Lancefield group B, type III (strain SS620), suspended in sterile hog gastric mucin. The enhancement of streptococcal virulence was measured by a significantly increased mortality in outbred ICR Swiss mice. An inbred C57BL6 strain of mice was resistant to the mucin-bacterial combination. Mucin, treated with Desferal to chelate the iron, did not lose the capacity to enhance the virulence of group B, type III streptococci in ICR Swiss mice. Iron-dextran was not a suitable substitute for mucin and failed to enhance the virulence of group B, type III streptococci. The results of these studies indicate that iron is not the resistance-lowering factor in this group B, type III streptococci-mucin model. ...
Stimulation of protective antibodies against type Ia and Ib group B streptococci by a type Ia polysaccharide-tetanus toxoid...Stimulation of protective antibodies against type Ia and Ib group B streptococci by a type Ia polysaccharide-tetanus toxoid...
Stimulation of protective antibodies against type Ia and Ib group B streptococci by a type Ia polysaccharide-tetanus toxoid conjugate vaccine: Infect.Immun.
Human group A streptococcal polysaccharide antibody ELISA Kit Prices,Sales - buy Human group A streptococcal polysaccharide...Human group A streptococcal polysaccharide antibody ELISA Kit Prices,Sales - buy Human group A streptococcal polysaccharide...
Where to buy Human group A streptococcal polysaccharide antibody ELISA Kit? Human group A streptococcal polysaccharide antibody ELISA Kit Suppliers,Human group A streptococcal polysaccharide antibody ELISA Kit Prices on Green Stone - medicinerawmaterials.com.
Streptococcus agalactiae in urinary tract infections. | Postgraduate Medical JournalStreptococcus agalactiae in urinary tract infections. | Postgraduate Medical Journal
Streptococcus agalactiae was found to be the cause of approximately 1% of urinary tract infections in a London teaching hospital in the 2 years studied. Of the forty-eight patients with this infection, forty-three were female. In nine patients the infection followed renal transplantation while in nine others it occurred in the presence of chronic renal failure. The rest, who included seven females who developed the infection following hysterectomies, had other clinical conditions which could have predisposed to such infections. The rarity of urinary tract infection by S. agalactiae is in contrast to the high frequency with which the organism colonizes the normal urethra. Serotypes III and II were the predominant isolates in these patients with urinary tract infections; this corresponds to the distribution of the different serotypes in the genito-urinary tract of normal individuals.. ...
Predicting outcomes of neonates born to GBS-positive women who received inadequate intrapartum antimicrobial prophylaxis<...Predicting outcomes of neonates born to GBS-positive women who received inadequate intrapartum antimicrobial prophylaxis<...
TY - JOUR. T1 - Predicting outcomes of neonates born to GBS-positive women who received inadequate intrapartum antimicrobial prophylaxis. AU - Kojima, Katsuaki. AU - Tanaka, Ryuma. AU - Nakajima, Keisuke. AU - Kurihara, Nobuyoshi. AU - Oba, Mari S aito. AU - Yamashita, Yukio. AU - Ishihara, Jun. AU - Yahagi, Naohisa. PY - 2014/5/1. Y1 - 2014/5/1. N2 - We determined the predicting factors of early-onset group B streptococcal (EOGBS) infection in neonates who were born to GBS carrier mothers with inadequate intrapartum antibiotic prophylaxis (IAP). Medical records of all neonates born from January 1, 2008 to April 1, 2010 were reviewed. Inadequate IAP was defined as delivery less than 4 hours (h) after the first administration of antimicrobial. Of 1910 neonates, 273 were born from mothers colonized with GBS, including 69 who received inadequate IAP. Of 69 neonates, nine showed symptoms, including respiratory distress, fever, tachycardia, vomiting, and irritability. Abnormalities in complete blood ...
Frontiers | CRISPR: A Useful Genetic Feature to Follow Vaginal Carriage of Group B Streptococcus | MicrobiologyFrontiers | CRISPR: A Useful Genetic Feature to Follow Vaginal Carriage of Group B Streptococcus | Microbiology
CRISPR (clustered regularly interspaced short palindromic repeats) and Cas (CRISPR-associated proteins) play a critical role in adaptive immunity against mobile genetic elements, especially phages, through their ability to acquire novel spacer sequences. Polarized spacer acquisition results in spacer polymorphism and temporal organization of CRISPR loci, making them attractive epidemiological markers. Group B Streptococcus (GBS), a genital commensal for 10 to 30% of healthy women and a major neonatal pathogen, possesses a ubiquitous and functional CRISPR1 locus. Our aim was to assess the CRISPR1 locus as an epidemiological marker to follow vaginal carriage of GBS in women. This study also allowed us to observe the evolution of the CRISPR1 locus in response to probable phage infection occurring in vivo.We followed carriage of GBS among 100 women over an eleven-year period, with a median duration of approximately two years. The CRISPR1 locus was highly conserved over time. The isolates that show the same
Analysis of Group B Streptococcal Isolates from Infants and Pregnant Women in Portugal Revealing Two Lineages with Enhanced...Analysis of Group B Streptococcal Isolates from Infants and Pregnant Women in Portugal Revealing Two Lineages with Enhanced...
Similar to previous studies, we found a diverse population among GBS colonization isolates, as well as among those causing invasive disease in Portugal, not only in terms of capsular polysaccharides, but also in genetic lineages defined by both PFGE and MLST (Table 1 and Fig. 1). Apart from serotypes VI and VIII, all other serotypes were found among our collection, and all except serotype VII were associated with both carriage and infection. Among the isolates causing infection, two isolates were identified expressing serotype IV, a serotype frequently associated with carriage in Asia (1) but infrequently found as a cause of neonatal infections in Western countries (29). Also noteworthy was the high prevalence of ST24 found exclusively among isolates of PFGE cluster A (n = 7/18 isolates characterized by MLST). ST24 was described in the publication proposing the GBS MLST scheme in a single isolate of serotype Ia (22) but has rarely been found among large collections of GBS isolates characterized ...
Group B streptococcal sepsis and congenital diaphragmatic hernia: do not overlook normal initial imaging studies! | ADC Fetal &...Group B streptococcal sepsis and congenital diaphragmatic hernia: do not overlook normal initial imaging studies! | ADC Fetal &...
You need to be signed in to access email alerts. If you have an account log in with your user name and password. If you dont have an account you can just enter your email address in the email box below ...
An outbreak of Streptococcus agalactiae infection in cage-cultured golden pompano, Trachinotus blochii (Lacépède), in Malaysia ...An outbreak of Streptococcus agalactiae infection in cage-cultured golden pompano, Trachinotus blochii (Lacépède), in Malaysia ...
Amal, M. N. A., Zamri-Saad, M., Iftikhar, A. R., Siti-Zahrah, A., Aziel, S. and Fahmi, S. (2012), An outbreak of Streptococcus agalactiae infection in cage-cultured golden pompano, Trachinotus blochii (Lacépède), in Malaysia. Journal of Fish Diseases, 35: 849-852. doi: 10.1111/j.1365-2761.2012.01443.x ...
ARISTOCILLIN 10 125MG - MedicScientist :: Total Health PortalARISTOCILLIN 10 125MG - MedicScientist :: Total Health Portal
Indications for Drugs ::. Bacterial infections, Endocarditis, Typhoid fever, Community-acquired pneumonia, Uncomplicated gonorrhea, Susceptible infections, Bacterial meningitis, Paratyphoid fever, Septicaemia, Intrapartum prophylaxis against group B streptococcal infections. Drug Dose ::. Adult: PO Susceptible infections 0.25-0.5 g 6 hrly. Typhoid and paratyphoid fever 1-2 g 6 hrly. Duration: 2 wk (acute infections); 4-12 wk (carriers). Uncomplicated gonorrhoea W/ probenecid: 2 g as single dose. IV Intrapartum prophylaxis against group B streptococcal infections Initial, 2 g, then 1 g 4 hrly until delivery. Septicaemia 150-200 mg/kg/day. Start with IV admin for 3 days, then IM inj 3-4 hrly. Continue treatment for at least 48-72 hr after patient has become asymptomatic or when there is evidence of bacterial eradication. IV/IM Susceptible infections 250-500 mg 6 hrly. Meningitis 2-3 g 4-6 hrly, may start w/ IV admin, then continue w/ IM inj. Inj Supplement to systemic therapy For ...
High prevalence of group B streptococcus ST17 hypervirulent clone among non-pregnant patients from a Hungarian venereology...High prevalence of group B streptococcus ST17 hypervirulent clone among non-pregnant patients from a Hungarian venereology...
Although Streptococcus agalactiae is the leading causative agent of neonatal sepsis and meningitis, recently it is increasingly isolated from non-pregnant adults. The relation between its presence in the genitourinary tract and manifested clinical symptoms of STD patients remains an open question. In this study, a complex epidemiological investigation of GBS isolates from a venerology clinic was performed. Ninety-six GBS isolates were serotyped and their genetic relatedness determined by PFGE. MLST was also performed for a subset of 20 isolates. The antibiotic susceptibility was tested with agar dilution. Surface proteins and the ST-17 hypervirulent clone was detected by PCR. The serotype prevalence was the following: V (29.2%), III (27.1%), Ia (22.9%), IV (10.4%), II (5.2%) and Ib (4.2%). A strong association was demonstrated between surface protein genes and serotypes. All isolates were fully susceptible to penicillin, but erythromycin and clindamycin resistance was high (41.7 and 35.4%, respectively)
Group B streptococcal meningitis has long-term effects on childrens developmental outcomes | Michigan MedicineGroup B streptococcal meningitis has long-term effects on children's developmental outcomes | Michigan Medicine
ANN ARBOR, Mich. - Parents of infants who survive bacterial meningitis caused by group B Streptococcus might have to live with the effects of the disease on their children long after theyre discharged from the hospital.. A new study in the journal Pediatrics finds that even though mortality rates of children infected with GBS meningitis have decreased in the past 25 years, just under half of children who survive the disease will suffer impairment as a result of the disease.. "These bacteria can quickly cause significant damage to the developing infant brain very quickly despite the infants having received excellent medical care," says Morven S. Edwards, M.D., professor of Pediatrics at Baylor College of Medicine and a co-author on the paper. "This is a potentially devastating illness and we still have a large percentage of infants who have poor outcomes after the infection.". According to the CDC, 25 percent of pregnant women carry GBS. It is routine for these women to receive antibiotics ...
Oliver Plumb, Author at Group B Strep Support - Page 3 of 4Oliver Plumb, Author at Group B Strep Support - Page 3 of 4
Dr Alison Bedford Russell described how clinical presentations of group B Strep were different between early-onset and late-onset. For example, group B Strep meningitis was more common in late-onset than early-onset GBS infection, while pneumonia was the reverse. Focal infection (infection in a particular area of the body) was "very,very rare" when it came to early-onset, and less rare for late-onset, for example, septic arthritis. Overall, generalised sepsis was the most common presentation. Dr Bedford Russell highlighted some of the difficulties of identifying early-onset GBS infection and also spoke of an increase in newborns presenting with "hypoxic ischemic encephalopathy" also referred to as HIE, a type of brain damage. The number of deaths as a result of group B Strep infection [...]. ...
Logic model-based failure analysis of intrapartum antibiotic prophylaxis (IAP) to prevent all cases of perinatal group B strep...Logic model-based failure analysis of intrapartum antibiotic prophylaxis (IAP) to prevent all cases of perinatal group B strep...
Dr. James A. McGregor has served as Professor of Obstetrics and Gynecology practicing at UCLA, USC and the University of Colorado Hospital. Dr. McGregor practiced as a fully engaged obstetrician and gynecologist for forty years at CedarsSinai/UCLA, Tucson Medical Center and University of Colorado Hospitals until his retirement in 2010. He is currently on Group B Strep Internationals Board of Directors and shares his expertise with GBSI through giving presentations and talking to providers and parents at perinatal conference exhibits around the world. ...
Beta Hemolytic Streptococcus Culture (Genital, Urine)Beta Hemolytic Streptococcus Culture (Genital, Urine)
This test looks for group B streptococcus (GBS) bacteria in a culture sample either from your urine or from secretions in your vagina and rectum.
Clinical analysis on 129 hospitalized children with purulent bacterial meningitis | International Journal of Current ResearchClinical analysis on 129 hospitalized children with purulent bacterial meningitis | International Journal of Current Research
Objective: The present study aimed to explore etiology, clinical features and treatment of children with purulent bacterial meningitis. Methodology: Hospital based retrospective cross sectional study, we reviewed cases of purulent bacterial meningitis occurring at Qilu hospital from January 2011 through September 2016. Records of all patients, comprising data on clinical presentations, laboratory findings and treatment were obtained and analyzed. Results: 129 children met criteria and were analyzed. Male to female ratio was 1.8:1, 82(63.6%) males and 47(36.4%) female. Cases were classified into five age groups: 1month-1 year76%, 1-2 years 4.7%, 2-5 years 8.5%, 5-12 years 8.5% and 12-15 years 2.3% (mean age is 2.93 months). Confirmed cases were 48(37%), the main bacteria cultured were Staphylococcal species 21%, Streptococcus pneumoniae 19%, group B Streptococcus (GBS) 15% and Escherichia coli 13%. Less detected bacteria were Enterococcus fecalis, Enterococcus fecium and Klebsiella pneumoniae. ...
NoteCategoriesNoteCategories
Babesiosis, Bacteremia, Bacterial Endocarditis Prevention (Bacterial Endocarditis Prophylaxis), Bacterial Infection, Bacterial Vaginitis, Bone infection (Osteomyelitis), Deep Neck Infection, Diverticulitis, Intraabdominal Infection, Joint Infection, Lemierres Syndrome, Pelvic Inflammatory Disease, Peritonitis, Pneumonia, Prevention of Perinatal Group B Streptococcal Disease, Sinusitis, Surgical Prophylaxis, Toxoplasmosis, Prophylaxis, ...
Approaches to Limit Intervention During Labor and Birth | AWHONN KansasApproaches to Limit Intervention During Labor and Birth | AWHONN Kansas
Obstetrician-gynecologists and other obstetric care providers should inform pregnant women with term premature rupture of membrane (PROM [also known as prelabor rupture of membranes]) who are considering a period of expectant care of the potential risks associated with expectant management and the limitations of available data. For informed women, if concordant with their individual preferences and if there are no other maternal or fetal reasons to expedite delivery, the choice of expectant management for a period of time may be appropriately offered and supported. For women who are group B streptococci (GBS) positive, however, administration of antibiotics for GBS prophylaxis should not be delayed while awaiting labor. In such cases, many patients and obstetrician-gynecologists or other obstetric care providers may prefer immediate induction ...
Streptococcus agalactiae - Wikipedia, a enciclopedia libreStreptococcus agalactiae - Wikipedia, a enciclopedia libre
A colonización por GBS normalmente non causa problemas en mulleres con boa saúde, non obstante durante o embarazo pode ás veces causar graves enfermidades na nai e no neonato. O GBS é a causa principal das infeccións neonatais bacterianas no meniño durante a xestación e despois do parto cunhas taxas de mortalidade significativas en nenos prematuros. As infeccións por GBS na nai poden causar corioamnionite (unha grave infección de tecidos placentarios infrecuente) e infeccións posparto. As infeccións do tracto urinario por GBS poden inducir o parto e causar un parto prematuro.[5] De feito, no mundo occidental, o GBS (en ausencia de medidas de prevención efectivas) é a principal causa de infeccións bacterianas graves de neonatos: septicemias, pneumonia e menixite, que poden causar a morte ou secuelas a longo prazo.[5] A infección neonatal por GBS orixínase normalmente no tracto reprodutor inferior de nais infectadas. As infección por GBS en neonatos son separadas en dúas ...
Million Settlement for Failure to Timely Diagnose Strep B Infection During Pregnancy | Sommers SchwartzMillion Settlement for Failure to Timely Diagnose Strep B Infection During Pregnancy | Sommers Schwartz
Sommers Schwartz attorneys Richard Groffsky and Lisa Esser secured a settlement with Cook County Health & Hospital System on behalf of a minor child and her mother. The lawsuit alleged that the defendants medical staff failed to timely diagnose a Strep B infection in the mother during her pregnancy, resulting in the baby contracting Strep B in utero and suffering serious birth injuries including septicemia, meningitis, retinal hemorrhage, and optic nerve damage. The complaint alleges that the health care providers who treated the pregnant mother violated the accepted standard of care by failing to submit the strep test to the pathology department within 24 to 48 hours after it was administered, which prevented the pregnant mother from being treated with antibiotics that would have protected the child from infection.. ...
Sepsis neonatal por Streptococcus Grupo BSepsis neonatal por Streptococcus Grupo B
CRUZ O, MAGDALENA; DOREN V, ADRIANA; TAPIA I, JOSÉ LUIS y ABARZUA C, FERNANDO. Group B Streptococcus neonatal sepsis: up-to-date. Rev. chil. pediatr. [online]. 2008, vol.79, n.5, pp.462-470. ISSN 0370-4106. http://dx.doi.org/10.4067/S0370-41062008000500003.. Group B Streptococcus is one of the leading bacterias causing early onset neonatal sepsis. It constitutes an important factor of neonatal morbidity and mortality and high costs in health. Many strategies have been formulated to avoid vertical transmission from the colonized mother to the newborn, in an attempt to prevent infection of the infant. The most used nowadays is antibiotic prophylaxis given to the mother during labor, depending on the results of recto-vaginal culture taken during 35 to 37 weeks of gestation. This strategy has importantly diminished the prevalence of early onset neonatal sepsis by this agent, although there is still concern about the potential generation of antibiotic resistance and drug-induced adverse reactions in ...
Literature - Abacus DiagnosticaLiterature - Abacus Diagnostica
Clinical evaluation of a molecular assay for direct and rapid screening for Streptococcus agalactiae in vaginal/rectal samples. Ataker F., Ahmad S., Lüthje P., Saeedi B. [Poster, Karolinska University Hospital, 2017, Sweden]. Comparative performance study of six commercial molecular assays for rapid detection of toxigenic Clostridium difficile. Paitan Y., Miller-Roll T., Adler A. Clin Microbiol Infect. 2017 Aug;23(8):567-572.. Evaluation of combined use of the MALDI-TOF and GenomEra MRSA/SA assay for the direct detection of methicillin resistance in Staphylococcus aureus from positive blood culture bottles. Pescador P., Romero-Gómez M.P., Gómez Gil R., Mingorance J. Enferm Infecc Microbiol Clin. 2016 Dec;34(10):687-688.. Direct detection of Streptococcus agalactiae using GenomEra CDX. Ghathian K., Dons L., Tapio A. [Poster, ECCMID, 2016, Netherlands]. Comparison of GenomEra C. difficile and Xpert C. difficile as Confirmatory Tests in a Multistep Algorithm for Diagnosis of Clostridium ...
Streptococcus Agalactiae - Office of Research Compliance | Montana State UniversityStreptococcus Agalactiae - Office of Research Compliance | Montana State University
Notify others working in the lab. Remove and don new PPE. Cover area of the spill with absorbent material and add 10 % Bleach. Allow 30 minutes hour of contact time. After 30 minutes and then cleanup and dispose of materials. ...
Permalien vers PbsP, a cell wall-anchored protein that binds plasminogen to promote hematogenous dissemination of group B...Permalien vers PbsP, a cell wall-anchored protein that binds plasminogen to promote hematogenous dissemination of group B...
Streptococcus agalactiae (Group B Streptococcus or GBS) is a leading cause of invasive infections in neonates whose virulence is dependent on its ability to
ORBi: Browsing ORBiORBi: Browsing ORBi
Poster (2014, May). Objectives Clearly associated to neonatal meningitis, Group B streptococci (GBS) classified as sequence type-17 (ST-17) are defined as the "highly virulent" clone amongst GBS. The aim of this study was to ... [more ▼]. Objectives Clearly associated to neonatal meningitis, Group B streptococci (GBS) classified as sequence type-17 (ST-17) are defined as the "highly virulent" clone amongst GBS. The aim of this study was to evaluate an easy and rapid method, recently described to detect ST-17 and ST-1 GBS, based on distinguishing peak-shifts present on the protein spectrum of these 2 sequence types, using a Microflex (Bruker) matrix-assisted laser desorption/ionization time of flight mass spectrometer (MALDI-TOF MS). Methods This study was performed on 67 multi locus sequence typed (MLST) GBS originated from the Belgian and Czech National Reference Centers, including 18 ST-17 and 16 ST-1. After culture on blood agar, an ethanol/formic acid extraction was performed on each ...
anti-Streptococcus agalactiae ZETA anticorps (FITC) (ABIN2913069)anti-Streptococcus agalactiae ZETA anticorps (FITC) (ABIN2913069)
Reactivité: Streptococcus agalactiae Hôte: Lapin Clone: Polyclonal Conjugué: FITC | Commandez ZETA lanticorps (ABIN2913069).
Team: Bacteria and Perinatality - Institut CochinTeam: Bacteria and Perinatality - Institut Cochin
Tazi A, Plainvert C, Anselem O, Ballon M, Marcou V, Seco A, El Alaoui F, Joubrel C, El Helali N, Falloukh E, Frigo A, Raymond J, Trieu-Cuot P, Branger C, Le Monnier A, Azria E, Ancel PY, Jarreau PH, Mandelbrot L, Goffinet F, Poyart C. Risk Factors for Infant Colonization by Hypervirulent CC17 Group B Streptococcus: Toward the Understanding of Late-onset Disease. Clin Infect Dis. 2019 Apr 4. pii: ciz033. doi: 10.1093/cid/ciz033.. (https://www.ncbi.nlm.nih.gov/pubmed/30946447). Weckel A, Ahamada D, Bellais S, Méhats C, Plainvert C, Longo M, Poyart C, Fouet A. The N-terminal domain of the R28 protein promotes emm28 Group A Streptococcus adhesion to host cells via direct binding to three integrins. J Biol Chem. (2018) 293:16006-16018. (https://www-ncbi-nlm-nih-gov.gate2.inist.fr/pubmed/30150299). Plainvert C, Longo M, Seringe E, Saintpierre B, Sauvage E, Ma L, Beghain J, Dmytruk N, Collobert G, Hernandez E, Manuel C, Astagneau P, Glaser P, Ariey F, Poyart C, Fouet A. A clone of the emergent ...
Recent advances in understanding the molecular basis of group B Streptococcus virulence. - PubMed - NCBIRecent advances in understanding the molecular basis of group B Streptococcus virulence. - PubMed - NCBI
PubMed comprises more than 30 million citations for biomedical literature from MEDLINE, life science journals, and online books. Citations may include links to full-text content from PubMed Central and publisher web sites.
Group B StreptococcusGroup B Streptococcus
Babies fed with breast milk containing a particular sugar are more likely to stay protected against severe bacterial infections. Breast milk; a liquid secreted from a mothers body immediately after childbirth, acts as a natural […] ...
How are infections caused by streptococcal bacteria treated? | Reference.comHow are infections caused by streptococcal bacteria treated? | Reference.com
Oral antibiotics are prescribed to treat strep throat caused by bacteria known as Streptococcus pyogenes, or group A streptococcus, states Mayo Clinic. A group B streptococcal infection is a...
STREP B: ENRICHED CULTURE MEDIUM TEST | Sir Nicholas Soames - Member of Parliament for Mid SussexSTREP B: ENRICHED CULTURE MEDIUM TEST | Sir Nicholas Soames - Member of Parliament for Mid Sussex
Sir Nicholass Question to the Department of Health onthe enriched culture medium test for group B streptococcus for pregnant women.
Hurt by Homebirth: Wrens story, on the 1st anniversary of his birth and deathHurt by Homebirth: Wren's story, on the 1st anniversary of his birth and death
In June last year, our son Daniel was born into the world. I like Tweeny was diagnosed as a strep b carrier after having a water infection too. There is major campaining in the UK as the strep b test is not offered routinely in the UK which it should be it was only the fact I had the water infection that it was picked up. The guidelines are that at anythim it is picked up there is no test between 35-37 weeks you should be given the iv antibiotics in labour. To that end thats what happened to me and Daniel was born after a fairly long labour but everything seemed fine. About 12 hours later the midwife noticed his breathing was too rapid and we were transferred by ambulance to a neonatal unit where it was diagnosed that the strep b had been passed on to him and he had pneumonia. It was only for the fact that they knew I was a strep b carrier that they observed him if not we would have been sent home. He was treated with iv antibiotics for 10 days and the infection cleared although initially it ...
Had a hysterectomy. Feeling of fatigue. On antibiotics, Strep B. Treatment and cause? Is this a side effect? - Doctors insight...Had a hysterectomy. Feeling of fatigue. On antibiotics, Strep B. Treatment and cause? Is this a side effect? - Doctor's insight...
Question - Had a hysterectomy. Feeling of fatigue. On antibiotics, Strep B. Treatment and cause? Is this a side effect?. Ask a Doctor about diagnosis, treatment and medication for Abdominal pain, Ask an OBGYN, Gynecologic Oncology
Missed Diagnosis of Strep B<...Missed Diagnosis of Strep B<...
You may have a medical negligence claim if you of your child have suffered due to a missed diagnosis of Strep B. Call us today on 0800 923 2079
The Alpha-like surface proteins: An example of an expanding family of adhesins<...The Alpha-like surface proteins: An example of an expanding family of adhesins<...
TY - JOUR. T1 - The Alpha-like surface proteins. T2 - An example of an expanding family of adhesins. AU - Creti, Roberta. AU - Baldassarri, L.. AU - Montanaro, L.. AU - Arciola, C. R.. PY - 2008/9. Y1 - 2008/9. N2 - The Alpha-like protein (Alp) family, repeat-containing surface proteins once thought to be important adhesion factors confined to pathogenic streptococci and enterococci, is broader than previously known. Analysis of the annotated microbial genomes has identified new potential members of the Alp family not only in other Gram- positive opportunistic pathogens but also in commensal microflora of the human gut and the skin. This finding has highlighted the importance of genome sequencing projects for unraveling in greater detail lateral gene transfer events involving virulence factors between pathogens and commensals. These should receive constant attention not only as part of infectious disease prevention programs, but also in the food and biotechnology industries.. AB - The Alpha-like ...
New hope for sufferers of urinary tract infection - Griffith NewsNew hope for sufferers of urinary tract infection - Griffith News
UTIs are among the most common infections in humans. In the USA alone, UTIs account for $3.5 billion in medical expenditure each year. Up to 40% of women have experienced at least one UTI episode in their lifetime. High rates of antibiotic resistance among bacteria that cause UTI is also a worrying trend.. The bacterium Streptococcus agalactiae causes 160,000 cases of UTI in the USA each year at a cost of $88 million to the healthcare system annually.. This reported incidence equates to 3 million cases globally and 12,000-56,000 in Australia annually. The bacterium also cause severe systemic and acute infections in other parts of the human body, and is a major cause of life-threatening neonatal infections.. The mechanisms by which streptococci cause UTI are not well understood. Dr Sullivans study characterised the role of a crucial control switch in the bacterium that determines that severity of UTI.. "Our work shows that this switch in the bacteria, termed CovR, is a master controller of how ...
Poster - Signs and symptoms of group B Strep infection in babiesPoster - Signs and symptoms of group B Strep infection in babies
Poster highlighting the most common signs and symptoms of group B Strep infection in babies, both of early-onset and late-onset GBS infection
My Baby Crazy Life: September 2012My Baby Crazy Life: September 2012
Another safe alternative treatment that can be used during labor is rinsing the vaginal and rectal areas with Hibiclens solution every 4-6 hours until baby is born. Hibiclens is the brand name for chlorohexidene, an antiseptic solution that is safe for both mother and baby yet very effective in reducing the incidence of GBS and E. coli infection in newborns. Unlike the herbal regimen recommended above, there have been specific scientific done which have proven the efficacy of this treatment (J Matern Fetal Neonatal Med 2002 Feb; 11 (2) :84-8 and British Journal of medicine 1997 Jul 26;3 15 (7102):216-9). This treatment can be used if the mother tests positive for GBS and her water breaks without labor or if other risk factors develop during labor. Your health care provider can answer any questions you may have about this safe and effective option for prevention of GBS infection in your baby. ...
Does the flu vaccine cause Guillain-Barré syndrome or not? | So much science, so little timeDoes the flu vaccine cause Guillain-Barré syndrome or not? | So much science, so little time
Guillain-Barré syndrome (GBS) is a pretty scary condition. It starts with weakness and tingling in the extremities and can eventually leading to paralysis. Although most people recover in time, death can occur. Luckily, its a rare disease. Its thought to result from an autoimmune process in which peripheral nerves are demyelinated and destroyed. What causes GBS? Infections…
Judiths Crusade: Daily Life with Cystic Fibrosis: Normal and NormalJudith's Crusade: Daily Life with Cystic Fibrosis: Normal and Normal
Today we took the little peanut for a follow up hearing test. She more than passed the standard newborn test before she was discharged from the NICU, but because she was on different antibiotics and because of the possible meningitis with the GBS infection we had to do a repeat to make sure the antibiotics didnt mess with her hearing. I know she hears fine, but this test helped confirm that - everything looks good! In some ways I wish we wouldve held off for another month and a half, to avoid the whole having to force her to sleep thing so they could check the nerve function leading to the brain stem. The tech explained that once babies are a year or older, they just do the tests we did before trying to get Judith to sleep. After an hour of Judith being very silly (and laughing at my mom for no reason - that was hilarious though!), she finally fell asleep and they were able to test 1 ear. We tried moving her head to get to the other ear, but she woke up, therefore we couldnt finish the test. ...
HypothiocyaniteHypothiocyanite
Hemolysis (microbiology)Hemolysis (microbiology)
Group B streptococcal infectionGroup B streptococcal infection
Neonatal meningitisNeonatal meningitis
CAMP testCAMP test
List of sequenced bacterial genomesList of sequenced bacterial genomes
Streptococcus pneumoniaeStreptococcus pneumoniae
GranadaeneGranadaene
SortaseSortase
Granada mediumGranada medium
Edmond NocardEdmond Nocard
TigecyclineTigecycline
Agar plateAgar plate
Pan-genomePan-genome
ErysipelasErysipelas
Diagnostic microbiologyDiagnostic microbiology
ThrombocytopeniaThrombocytopenia
Community-acquired pneumoniaCommunity-acquired pneumonia
CefodizimeCefodizime
ChildbirthChildbirth
Keratin 4Keratin 4
Growth mediumGrowth medium
EndometritisEndometritis
Overwhelming post-splenectomy infectionOverwhelming post-splenectomy infection
Bacterial capsuleBacterial capsule
Mastitis in dairy cattleMastitis in dairy cattle
HydrotaeaHydrotaea
Reactive lymphocyteReactive lymphocyte
Neonatal infectionNeonatal infection
Ramaria botrytisRamaria botrytis
Staphylococcal scalded skin syndromeStaphylococcal scalded skin syndrome
Celulitis - Wikipedija, prosta enciklopedijaCelulitis - Wikipedija, prosta enciklopedija
Urinary tract infectionUrinary tract infection
Growth mediumGrowth medium
StreptococcusStreptococcus
BacillusBacillus
Panxenoma, a enciclopedia librePanxenoma, a enciclopedia libre
Gas gangreneGas gangrene
Streptokokk - VikipeediaStreptokokk - Vikipeedia
Bacterial capsuleBacterial capsule
TedizolidTedizolid
List of infectious diseasesList of infectious diseases
StreptococcusStreptococcus
List of parasites of humansList of parasites of humans
Streptococcus agalactiae, a enciclopedia libreStreptococcus agalactiae, a enciclopedia libre
Streptococcus bahasa Indonesia, ensiklopedia bebasStreptococcus bahasa Indonesia, ensiklopedia bebas
Gram positive, ang malayang ensiklopedyaGram positive, ang malayang ensiklopedya
Streptococcus, ang malayang ensiklopedyaStreptococcus, ang malayang ensiklopedya
Scarlet feverScarlet fever
AmikacinAmikacin
腦膜炎 - 維基百科，自由的百科全書腦膜炎 - 維基百科，自由的百科全書
Imipenem/cilastatinImipenem/cilastatin
Streptococcus agalactiaeStreptococcus agalactiae
Liosta de ghalair thógálacha - VicipéidLiosta de ghalair thógálacha - Vicipéid
ClarithromycinClarithromycin
StreptokoketStreptokoket
Streptococcus anginosus groupStreptococcus anginosus group
AnatomyOrganismsDiseasesChemicals and DrugsAnalytical, Diagnostic and Therapeutic Techniques and EquipmentPhenomena and ProcessesDisciplines and OccupationsAnthropology, Education, Sociology and Social PhenomenaTechnology, Industry, AgricultureInformation ScienceNamed GroupsHealth CareGeographicals
Streptococcus agalactiaeStreptococcusStreptococcal InfectionsStreptococcus pyogenesStreptococcus pneumoniaeStreptococcus mutansMastitis, BovineSerotypingBacterial ProteinsMastitisStreptococcus PhagesMeningitis, BacterialErythromycinStreptococcus suisDNA, BacterialAnti-Bacterial AgentsStreptococcus bovisBacterial AdhesionVaginaStreptococcus mitisBacterial CapsulesMicrobial Sensitivity TestsStreptococcus equiGenes, BacterialMolecular Sequence DataStreptococcus oralisDrug Resistance, BacterialVirulenceProphagesAntigens, BacterialStreptococcus sobrinusAminoacyltransferasesSequence Analysis, DNABacterial Typing TechniquesLincosamidesEnterococcus faecalisGram-Positive BacteriaBacteriological TechniquesCulture MediaClindamycinVirulence FactorsCattleKeratin-4Adhesins, BacterialPneumococcal InfectionsStreptococcus gordoniiGene Expression Regulation, BacterialElectrophoresis, Gel, Pulsed-FieldTeichoic AcidsPyometraAmobarbitalTetracycline ResistanceRandom Amplified Polymorphic DNA TechniqueStreptococcus thermophilusGenome, BacterialHemolysin ProteinsInfant, NewbornPolysaccharides, BacterialFasciitis, NecrotizingLactoperoxidaseArcanobacteriumPolymerase Chain ReactionMacrolidesMilkDNA FingerprintingProkaryotic Initiation Factor-2Carrier StateMycoplasma InfectionsReceptors, FibrinogenAntibodies, BacterialInterspersed Repetitive SequencesDNA Transposable ElementsMeningitisPhylogenyBase SequencePolysaccharide-LyasesGenetic VariationAmino Acid SequenceMolecular TypingZimbabwePigments, BiologicalPregnancy Complications, InfectiousTetracyclineDrug Resistance, MicrobialBacteriaDairyingArthritis, InfectiousStaphylococcusMultilocus Sequence TypingCell WallGene OrderMouthStaphylococcus aureusHemolysisColony Count, MicrobialGenotypeStreptolysinsRectumPregnancyFimbriae, BacterialSpecies SpecificityEnterococcusCattle DiseasesGram-Negative BacteriaAnti-Infective AgentsStreptococcus intermediusGenomic IslandsConjugation, GeneticPharyngitisGene Transfer, HorizontalBloodDental PlaqueBacteremiaPneumonia, PneumococcalMolecular EpidemiologyCluster AnalysisMycoplasmaFibrinogenStreptococcal VaccinesRNA, BacterialMutagenesis, InsertionalGene DeletionEscherichia coliPenicillin ResistanceFimbriae ProteinsCloning, MolecularSheep DiseasesOperonGram-Positive Bacterial InfectionsSalivaPharynx
Streptococcus agalactiae - WikipediaStreptococcus agalactiae - Wikipedia
Streptococcus agalactiaeStreptococcus agalactiae
Typing of Streptococcus agalactiae (Lancefield group B) | SpringerLinkTyping of Streptococcus agalactiae (Lancefield group B) | SpringerLink
Streptococcus agalactiae Lehmann and Neumann ATCC ® BAA-611D-5&traStreptococcus agalactiae Lehmann and Neumann ATCC ® BAA-611D-5&tra
Streptococcus agalactiae Srr-1 protein Summary Report | CureHunterStreptococcus agalactiae Srr-1 protein Summary Report | CureHunter
Genome analysis of multiple pathogenic isolates of Streptococcus agalactiae: implications for the microbial pan-genome | CBCBGenome analysis of multiple pathogenic isolates of Streptococcus agalactiae: implications for the microbial 'pan-genome' | CBCB
Detection and quantification of Streptococcus agalactiae from aquaculture with molecular means - Enlighten: ThesesDetection and quantification of Streptococcus agalactiae from aquaculture with molecular means - Enlighten: Theses
Enhanced Expression of lmb Gene Encoding Laminin-Binding Protein in Streptococcus agalactiae Strains Harboring IS1548 in scpB...Enhanced Expression of lmb Gene Encoding Laminin-Binding Protein in Streptococcus agalactiae Strains Harboring IS1548 in scpB...
A Comparative Investigation of Streptococcus Agalactiae Isolates from Fish and CattleA Comparative Investigation of Streptococcus Agalactiae Isolates from Fish and Cattle
Human Streptococcus agalactiae strains in aquatic mammals and fish | BMC Microbiology | Full TextHuman Streptococcus agalactiae strains in aquatic mammals and fish | BMC Microbiology | Full Text
Which Of The Following Statements About Group B A Streptococcus (Streptococcus Agalactiae) Is Not Correct?Which Of The Following Statements About Group B A Streptococcus (Streptococcus Agalactiae) Is Not Correct?
Complete genome sequence of serotype III Streptococcus agalactiae sequence type 17 strain 874391. - PacBioComplete genome sequence of serotype III Streptococcus agalactiae sequence type 17 strain 874391. - PacBio
Streptococcus agalactiae | Pathway MedicineStreptococcus agalactiae | Pathway Medicine
Datapunk | SUBSTRATA Substrata: Streptococcus agalactiaeDatapunk | SUBSTRATA Substrata: Streptococcus agalactiae
Streptococcus Laboratory: Streptococcus agalactiae | CDCStreptococcus Laboratory: Streptococcus agalactiae | CDC
Streptococcus agalactiae serotype III (strain NEM316)Streptococcus agalactiae serotype III (strain NEM316)
Streptococcus agalactiae MC633, whole genome shotgun sequence - Nucleotide - NCBIStreptococcus agalactiae MC633, whole genome shotgun sequence - Nucleotide - NCBI
Streptococcus agalactiae Lehmann and Neumann ATCC ® 12386™Streptococcus agalactiae Lehmann and Neumann ATCC ® 12386™
KEGG GENOME: Streptococcus agalactiae ILRI005KEGG GENOME: Streptococcus agalactiae ILRI005
Streptococcus agalactiae MC632 Contig248, whole genome shotgun sequenc - Nucleotide - NCBIStreptococcus agalactiae MC632 Contig248, whole genome shotgun sequenc - Nucleotide - NCBI
Streptococcus agalactiae - Wikipedia, a enciclopedia libreStreptococcus agalactiae - Wikipedia, a enciclopedia libre
Streptococcus Agalactiae - Office of Research Compliance | Montana State UniversityStreptococcus Agalactiae - Office of Research Compliance | Montana State University
Streptococcus agalactiae Meningitis in Adult Patient: A Case Report and Literature ReviewStreptococcus agalactiae Meningitis in Adult Patient: A Case Report and Literature Review
What are the risk factors for Streptococcus agalactiae (group B Streptococcus) (GBS) meningitis in adults?What are the risk factors for Streptococcus agalactiae (group B Streptococcus) (GBS) meningitis in adults?
Streptococcus agalactiae : Wikis (The Full Wiki)Streptococcus agalactiae : Wikis (The Full Wiki)
Streptococcus agalactiae serotype V (strain ATCC BAA-611 / 2603 V/R)Streptococcus agalactiae serotype V (strain ATCC BAA-611 / 2603 V/R)
KEGG PATHWAY: Metabolic pathways - Streptococcus agalactiae 2603 (serotype V)KEGG PATHWAY: Metabolic pathways - Streptococcus agalactiae 2603 (serotype V)
Genome analysis of multiple pathogenic isolates of Streptococcus agalactiae: Implications for the microbial pan-genome | PNASGenome analysis of multiple pathogenic isolates of Streptococcus agalactiae: Implications for the microbial "pan-genome" | PNAS
Streptococcus agalactiae Septic Arthritis of the Shoulder and the Sacroiliac Joints: A Case ReportStreptococcus agalactiae Septic Arthritis of the Shoulder and the Sacroiliac Joints: A Case Report
Streptococcus agalactiae | Infection and ImmunityStreptococcus agalactiae | Infection and Immunity
Streptococcus agalactiae in urinary tract infections. | Postgraduate Medical JournalStreptococcus agalactiae in urinary tract infections. | Postgraduate Medical Journal
Streptococcus Agalactiae Antibody (224/46) (NB100-62933): Novus BiologicalsStreptococcus Agalactiae Antibody (224/46) (NB100-62933): Novus Biologicals
Characterization and antibiotic susceptibility of Streptococcus agalactiae isolates causing urinary tract infections. | Sigma...Characterization and antibiotic susceptibility of Streptococcus agalactiae isolates causing urinary tract infections. | Sigma...
An outbreak of Streptococcus agalactiae infection in cage-cultured golden pompano, Trachinotus blochii (Lacépède), in Malaysia ...An outbreak of Streptococcus agalactiae infection in cage-cultured golden pompano, Trachinotus blochii (Lacépède), in Malaysia ...
The Clinical and Molecular Epidemiology of Streptococcus Agalactiae Colonisation on the Kenyan Coast - Full Text View -...The Clinical and Molecular Epidemiology of Streptococcus Agalactiae Colonisation on the Kenyan Coast - Full Text View -...
RCSB PDB - 2OGI: Crystal structure of a putative metal dependent phosphohydrolase (sag1661) from streptococcus agalactiae...RCSB PDB - 2OGI: Crystal structure of a putative metal dependent phosphohydrolase (sag1661) from streptococcus agalactiae...
KEGG PATHWAY: Glycolysis / Gluconeogenesis - Streptococcus agalactiae ILRI005KEGG PATHWAY: Glycolysis / Gluconeogenesis - Streptococcus agalactiae ILRI005
Eradication of Streptococcus agalactiae mastitis from a 30-cow dairy herdEradication of Streptococcus agalactiae mastitis from a 30-cow dairy herd
AnatomyOrganismsDiseasesChemicals and DrugsAnalytical, Diagnostic and Therapeutic Techniques and EquipmentPhenomena and ProcessesDisciplines and OccupationsAnthropology, Education, Sociology and Social PhenomenaTechnology, Industry, AgricultureInformation ScienceNamed GroupsHealth CareGeographicals
Streptococcus agalactiaeStreptococcusStreptococcal InfectionsStreptococcus pyogenesStreptococcus pneumoniaeStreptococcus mutansMastitis, BovineSerotypingBacterial ProteinsMastitisStreptococcus PhagesMeningitis, BacterialErythromycinStreptococcus suisDNA, BacterialAnti-Bacterial AgentsStreptococcus bovisBacterial AdhesionVaginaStreptococcus mitisBacterial CapsulesMicrobial Sensitivity TestsStreptococcus equiGenes, BacterialMolecular Sequence DataStreptococcus oralisDrug Resistance, BacterialVirulenceProphagesAntigens, BacterialStreptococcus sobrinusAminoacyltransferasesSequence Analysis, DNABacterial Typing TechniquesLincosamidesEnterococcus faecalisGram-Positive BacteriaBacteriological TechniquesCulture MediaClindamycinVirulence FactorsCattleKeratin-4Adhesins, BacterialPneumococcal InfectionsStreptococcus gordoniiGene Expression Regulation, BacterialElectrophoresis, Gel, Pulsed-FieldTeichoic AcidsPyometraAmobarbitalTetracycline ResistanceRandom Amplified Polymorphic DNA TechniqueStreptococcus thermophilusGenome, BacterialHemolysin ProteinsInfant, NewbornPolysaccharides, BacterialFasciitis, NecrotizingLactoperoxidaseArcanobacteriumPolymerase Chain ReactionMacrolidesMilkDNA FingerprintingProkaryotic Initiation Factor-2Carrier StateMycoplasma InfectionsReceptors, FibrinogenAntibodies, BacterialInterspersed Repetitive SequencesDNA Transposable ElementsMeningitisPhylogenyBase SequencePolysaccharide-LyasesGenetic VariationAmino Acid SequenceMolecular TypingZimbabwePigments, BiologicalPregnancy Complications, InfectiousTetracyclineDrug Resistance, MicrobialBacteriaDairyingArthritis, InfectiousStaphylococcusMultilocus Sequence TypingCell WallGene OrderMouthStaphylococcus aureusHemolysisColony Count, MicrobialGenotypeStreptolysinsRectumPregnancyFimbriae, BacterialSpecies SpecificityEnterococcusCattle DiseasesGram-Negative BacteriaAnti-Infective AgentsStreptococcus intermediusGenomic IslandsConjugation, GeneticPharyngitisGene Transfer, HorizontalBloodDental PlaqueBacteremiaPneumonia, PneumococcalMolecular EpidemiologyCluster AnalysisMycoplasmaFibrinogenStreptococcal VaccinesRNA, BacterialMutagenesis, InsertionalGene DeletionEscherichia coliPenicillin ResistanceFimbriae ProteinsCloning, MolecularSheep DiseasesOperonGram-Positive Bacterial InfectionsSalivaPharynx
StrainsInfectionNeonatesInvasiveSpeciesSerotype III Streptococcus agalactiaeGroupCharacterizationMastitisStrainBacteriumTaxonomyHemolyticNewbornGenomeProteinComplete GenomePathogenInfectionSepsisMeningitisAnti-Streptococcus agalactiaeMastitisAntibodyGram-positiveA909SequencePenicillinCharacterizationVaginalBiosynthesisPathogenicDiseaseAntimicrobial resistanceGene
Strains5
Infection1
Neonates1
Invasive1
Species1
Serotype III Streptococcus agalactiae1
Group20
Characterization1
Mastitis1
  • Streptococcus agalactiae also causes bovine mastitis and the organism can be isolated from milk samples. (auburn.edu)
Strain2
Bacterium1
  • Streptococcus agalactiae is the causative bacterium of streptococcosis and causes severe economic losses in wild and cultured fish and cattle, worldwide. (auburn.edu)
Taxonomy1
  • Brown, J.H. Classification of streptococci, groups A, B, C and D. International Bulletin of Bacteriological Nomenclature and Taxonomy 1953, 3: 163-169. (springer.com)
Hemolytic1
Newborn1
Genome1
Protein1
Complete Genome2
Pathogen12
Infection8
Sepsis7
Meningitis7
Anti-Streptococcus agalactiae1
Mastitis3
Antibody3
Gram-positive1
A9091
Sequence4
Penicillin1
Characterization1
Vaginal1
Biosynthesis2
  • Although the structure of this complex carbohydrate antigen has been solved, little is known of its biosynthesis beyond the identification of a relevant locus in sequenced S. agalactiae genomes. (brad.ac.uk)
  • Genomic analysis has allowed us to predict the pathway leading to the biosynthesis of GBC of S. agalactiae. (brad.ac.uk)
Pathogenic1
Disease3
  • The results of this study were presented in part at the 1st International Symposium on Streptococcus agalactiae Disease, Cape Town, South Africa, February 20-23, 2018. (ajtmh.org)
  • If S. agalactiae can be detected in sick fish within 12 h it is possible to pre-emptively treat the remaining fish with an oral antibiotic before the disease begins to cause mass mortalities. (gla.ac.uk)
  • Fish isolates were evaluated to determine whether a single clone or multi-clones of S. agalactiae were responsible for streptococcal disease in mullet and seabream during the Kuwait Bay, Kuwait epidemic by amplified fragment length polymorphism (AFLP). (auburn.edu)
Antimicrobial resistance1
Gene5
Genome analysis of multiple pathogenic isolates of Streptococcus agalactiae: Implications for the microbial pan-genome | PNAS
Genome analysis of multiple pathogenic isolates of Streptococcus agalactiae: Implications for the microbial "pan-genome" | PNAS (pnas.org)
Detection and quantification of Streptococcus agalactiae from aquaculture with molecular means - Enlighten: Theses
Detection and quantification of Streptococcus agalactiae from aquaculture with molecular means - Enlighten: Theses (theses.gla.ac.uk)
A hemolytic pigment of Group B Streptococcus allows bacterial penetration of human placenta | JEM
A hemolytic pigment of Group B Streptococcus allows bacterial penetration of human placenta | JEM (jem.rupress.org)
Streptococcus agalactiae - Wikipedia, a enciclopedia libre
Streptococcus agalactiae - Wikipedia, a enciclopedia libre (gl.wikipedia.org)
Streptococcus agalactiae : Wikis (The Full Wiki)
Streptococcus agalactiae : Wikis (The Full Wiki) (thefullwiki.org)
Superoxide Dismutase and Catalase Do Not Affect the Pulmonary Hypertensive Response to Group B Streptococcus in the Lamb |...
Superoxide Dismutase and Catalase Do Not Affect the Pulmonary Hypertensive Response to Group B Streptococcus in the Lamb |... (nature.com)
IUCr) Acta Crystallographica Section D Volume 70, Part 11, November 2014
IUCr) Acta Crystallographica Section D Volume 70, Part 11, November 2014 (journals.iucr.org)
Jélinková 3139 Strain Passport - StrainInfo
Jélinková 3139 Strain Passport - StrainInfo (straininfo.net)
Pathogen biology (12 entities) | Institut Pasteur
Pathogen biology (12 entities) | Institut Pasteur (pasteur.fr)
Novel IgG-Degrading Enzymes of the IgdE Protease Family Link Substrate Specificity to Host Tropism of Streptococcus Species
Novel IgG-Degrading Enzymes of the IgdE Protease Family Link Substrate Specificity to Host Tropism of Streptococcus Species (journals.plos.org)
Genomics of Pneumonia and Meningitis (and neonatal sepsis) - Wellcome Sanger Institute
Genomics of Pneumonia and Meningitis (and neonatal sepsis) - Wellcome Sanger Institute (sanger.ac.uk)
Zithromax (Azithromycin): Side Effects, Interactions, Warning, Dosage & Uses
Zithromax (Azithromycin): Side Effects, Interactions, Warning, Dosage & Uses (rxlist.com)
Cleocin - FDA prescribing information, side effects and uses
Cleocin - FDA prescribing information, side effects and uses (drugs.com)
Zosyn (Piperacillin and Tazobactam Injection): Side Effects, Interactions, Warning, Dosage & Uses
Zosyn (Piperacillin and Tazobactam Injection): Side Effects, Interactions, Warning, Dosage & Uses (rxlist.com)
Noroxin (Norfloxacin): Side Effects, Interactions, Warning, Dosage & Uses
Noroxin (Norfloxacin): Side Effects, Interactions, Warning, Dosage & Uses (rxlist.com)
Cefpodoxime Proxetil - FDA prescribing information, side effects and uses
Cefpodoxime Proxetil - FDA prescribing information, side effects and uses (drugs.com)
Cefepime - FDA prescribing information, side effects and uses
Cefepime - FDA prescribing information, side effects and uses (drugs.com)
Ceftriaxone - FDA prescribing information, side effects and uses
Ceftriaxone - FDA prescribing information, side effects and uses (drugs.com)
Macrodantin (Nitrofurantoin Macrocystals Capsule): Side Effects, Interactions, Warning, Dosage & Uses
Macrodantin (Nitrofurantoin Macrocystals Capsule): Side Effects, Interactions, Warning, Dosage & Uses (rxlist.com)
Suprax (Cefixime): Side Effects, Interactions, Warning, Dosage & Uses
Suprax (Cefixime): Side Effects, Interactions, Warning, Dosage & Uses (rxlist.com)
Ceftin (Cefuroxime Axetil): Uses, Dosage, Side Effects, Interactions, Warning
Ceftin (Cefuroxime Axetil): Uses, Dosage, Side Effects, Interactions, Warning (rxlist.com)
Linezolid Oral Suspension - FDA prescribing information, side effects and uses
Linezolid Oral Suspension - FDA prescribing information, side effects and uses (drugs.com)
Iquix - FDA prescribing information, side effects and uses
Iquix - FDA prescribing information, side effects and uses (drugs.com)
The Clinical and Molecular Epidemiology of Streptococcus Agalactiae Colonisation on the Kenyan Coast - Full Text View -...
The Clinical and Molecular Epidemiology of Streptococcus Agalactiae Colonisation on the Kenyan Coast - Full Text View -... (clinicaltrials.gov)
Characterization and antibiotic susceptibility of Streptococcus agalactiae isolates causing urinary tract infections. | Sigma...
Characterization and antibiotic susceptibility of Streptococcus agalactiae isolates causing urinary tract infections. | Sigma... (sigmaaldrich.com)
Search
Search (cambridge.org)
IUCr) Acta Crystallographica Section D Volume 63, Part 6, June 2007
IUCr) Acta Crystallographica Section D Volume 63, Part 6, June 2007 (journals.iucr.org)
Antibiotics | Free Full-Text | Update on Acute Bone and Joint Infections in Paediatrics: A Narrative Review on the Most Recent...
Antibiotics | Free Full-Text | Update on Acute Bone and Joint Infections in Paediatrics: A Narrative Review on the Most Recent... (mdpi.com)
Perlakuan Celup Puting setelah Pemerahan terhadap Keberadaan Bakteri Patogen, Staphylococcus aureus, Streptococcus agalactiae,...
Perlakuan Celup Puting setelah Pemerahan terhadap Keberadaan Bakteri Patogen, Staphylococcus aureus, Streptococcus agalactiae,... (jurnal.ugm.ac.id)
DailyMed - CLARITHROMYCIN tablet, film coated, extended release
DailyMed - CLARITHROMYCIN tablet, film coated, extended release (dailymed.nlm.nih.gov)
Cellulitis | 5-Minute Clinical Consult
Cellulitis | 5-Minute Clinical Consult (unboundmedicine.com)