Skin Physiological Phenomena
Digestive System and Oral Physiological Phenomena
Reproductive and Urinary Physiological Phenomena
Musculoskeletal and Neural Physiological Phenomena
Circulatory and Respiratory Physiological Phenomena
Integumentary System Physiological Phenomena
Sports Nutritional Physiological Phenomena
Reproductive Physiological Phenomena
Physiological Phenomena
Elder Nutritional Physiological Phenomena
Urinary Tract Physiological Phenomena
Skin
Digestive System Physiological Phenomena
Skin Aging
Ocular Physiological Phenomena
Nervous System Physiological Phenomena
Respiratory Physiological Phenomena
Plant Physiological Phenomena
Skin Tests
Cardiovascular Physiological Phenomena
Maternal Nutritional Physiological Phenomena
Nutritional Physiological Phenomena
Infant Nutritional Physiological Phenomena
Skin, Artificial
Raynaud Disease
Temperature regulation and heat dissipation during flight in birds. (1/1118)
Core and skin temperature were measured by radiotelemetry in starlings (Sturnus vulgaris) during 30 min flights in a wind tunnel. Core temperature was independent of ambient temperature from 0 to 28 degrees C. The temporal mean of the monitored core temperature during flight was 42-7 degrees C in one bird and 44-0 degrees C in another. These temperatures are 2-4 degrees C higher than the resting temperature in starlings, and are among the highest steady-state temperatures observed in any animal. Skin temperature on the breast was within a few degrees of core temperature. In some locations skin temperature was higher at low ambient temperatures than at intermediate ambient temperatures. An analysis of the data shows that a high core temperature does not function as an aid to head dissipation. On the contrary, insulation is adjusted to maintain a high temperature, presumably because it is necessary for flight. The increase in skin temperature at low ambient temperatures is believed to be a result of a decrease in heat flow through the breast feathers brought about by feather adjustments, to compensate for an unavoidable increase in heat flow in unfeathered or poorly feathered parts of the body. (+info)Topical gene delivery to murine skin. (2/1118)
We topically applied naked plasmid DNA containing the luciferase or chloramphenicol acetyltransferase cDNA directly to mouse skin. Gene expression was detected in skin samples as early as 4 h after DNA application, plateaued from 16 to 72 h post-application, and had decreased significantly by 7 d post-application. Reporter gene activity following topical DNA delivery was comparable with that produced by intradermal injection of DNA. Plasmid DNA at concentrations > or =0.25 microg per microl were required to achieve maximal expression levels. Reporter gene expression following topical administration was largely confined to the superficial layers of the epidermis and to hair follicles. Surprisingly, certain cationic liposomes inhibited the efficiency of cutaneous gene transfer. This technique provides a simple, clinically relevant approach to deliver genes to the skin, with potential application in treating a variety of cutaneous disorders. (+info)UVA exposure of human skin reconstructed in vitro induces apoptosis of dermal fibroblasts: subsequent connective tissue repair and implications in photoaging. (3/1118)
The skin reconstructed in vitro has been previously shown to be a useful model to investigate the effects of UVB exposure (Bernerd and Asselineau, 1997). The present study describes the response to UVA irradiation. Major alterations were observed within the dermal compartment. Apoptosis of fibroblasts located in the superficial area of the dermal equivalent was observed as soon as 6 h after irradiation, leading to their disappearance after 48 h. This effect was obtained without major alterations of epidermal keratinocytes suggesting a differential cell type sensitivity to UVA radiations. In addition, collagenase I was secreted by dermal fibroblasts. The UVA dermal effects could be observed even after removal of the epidermis during the post irradiation period, demonstrating that they were independent of the keratinocyte response. The analysis of the tissue regeneration during the following 2 weeks revealed a connective tissue repair via fibroblasts proliferation, migration and active synthesis of extracellular matrix proteins such as fibronectin and procollagen I. This cellular recolonization of the superficial part of the dermal equivalent was due to activation of surviving fibroblasts located deeply in the dermal equivalent. The direct damage in the dermis and the subsequent connective tissue repair may contribute to the formation of UVA-induced dermal alterations. (+info)Time-dependent ultrastructural changes to porcine stratum corneum following an electric pulse. (4/1118)
The morphological changes to heat-stripped porcine stratum corneum following an electroporating pulse were studied by time-resolved freeze fracture electron microscopy. Pulses at a supra-electroporation threshold of 80 volts and 300 microseconds were applied across the stratum corneum with a pair of copper plate electrodes, which also served as cooling contacts. Multilamellar vesicles of 0.1-5.5 mm in diameter in the intercellular lipid bilayers of the stratum corneum appeared in less than milliseconds after pulsing. Pulsed samples exhibited aggregations of vesicles, whereas only occasional single vesicles were seen in the unpulsed samples. Aggregates form in less than a millisecond and disappear within minutes after the pulse. Their size ranged from 0.3 to 700 mm2. The size of individual vesicles, aggregate density, and size were analyzed as functions of postpulse time. These aggregate formations seem to be a secondary reaction to the pulse-induced skin permeabilization, determined by the resistance drop and recovery after the pulse. Heating the samples to 65 degrees C also caused vesicle aggregates of similar appearance to form, suggesting that these aggregations are related to the heating effect of the pulse. Hydration is thought to play an important role in aggregate formation. (+info)Keratinocyte growth regulation in fibroblast cocultures via a double paracrine mechanism. (5/1118)
Epithelial-mesenchymal interactions play an important role in regulating tissue homeostasis and repair. For skin, the regulatory mechanisms of epidermal-dermal interactions were studied in cocultures of normal human epidermal keratinocytes (NEK) and dermal fibroblasts (HDF) rendered postmitotic by alpha-irradiation (HDFi). The expression kinetics of different cytokines and their receptors with presumed signalling function in skin were determined at the RNA and protein level in mono- and cocultured NEK and HDFi. In cocultured HDFi, mRNA and protein synthesis of keratinocyte growth factor (KGF) (FGF-7) was strongly enhanced, whereas in cocultured keratinocytes interleukin (IL)-1alpha and -1beta mRNA expression increased compared to monocultures. Thus we postulated that IL-1, which had no effect on keratinocyte proliferation, induced in fibroblasts the expression of factors stimulating keratinocyte proliferation, such as KGF. The functional significance of this reciprocal modulation was substantiated by blocking experiments. Both IL-1alpha and -1beta-neutralizing antibodies and IL-1 receptor antagonist significantly reduced keratinocyte proliferation supposedly through abrogation of KGF production, because IL-1 antibodies blocked the induced KGF production. These data indicate a regulation of keratinocyte growth by a double paracrine mechanism through release of IL-1 which induces KGF in cocultured fibroblasts. Thus IL-1, in addition to its proinflammatory function in skin, may play an essential role in regulating tissue homeostasis. (+info)Injection of pre-psoriatic skin with CD4+ T cells induces psoriasis. (6/1118)
Psoriasis is an immunologically mediated skin disease linked to several different class I major histocompatibility complex alleles. However, the phenotype of the pathogenic lymphocyte and nature of the T cell activating event which triggers conversion of symptomless (PN) skin into psoriatic plaques (PP skin) is unknown. This study extends our previous observations in which autologous blood-derived immunocytes were injected into PN skin engrafted onto SCID mice to produce full-fledged PP lesions. The first question addressed is whether injected CD4+ T cells or CD8+ T cells were responsible for phenotypic conversion of PN to PP skin. In five different patients only CD4+ but not CD8+ T cell lines produced psoriatic lesions. Next, immunological events occurring within PN skin following injection of CD4+ T cells in grafts that had sufficient tissue available for detailed analysis was examined. In two patients, intraepidermal resident CD8+ T cells were induced to proliferate during lesion development, expressing acute activation markers CD25 and CD69. In another patient, injection of CD4+ T cells revealed CD69 expression by intraepidermal CD4+ as well as CD8+ T cells. To explore the molecular basis for local T cell activation and proliferation, we discovered that intraepidermal immunocytes, including both CD4 and CD8+ T cells, expressed surface receptors (ie, CD94, CD158a, CD158b) typically confined to natural killer cells (ie, natural killer receptors; NKRs) accumulated immediately before onset of acute lesions. The presence of NKR bearing immunocytes was also observed in 10 of 15 different biopsies of chronic plaques taken directly from patients, whereas PN skin (n = 8) or normal skin from healthy donors (n = 8), did not contain such NKR positive immunocytes. Of particular relevance to psoriasis is that these NKRs recognize various class I alleles including those typically inherited by psoriatic family members such as HLA-C and HLA-B allotypes. We conclude that injection of CD4+ T cells into PN skin triggers a series of local immunologically mediated stimulatory events that produce further T cell activation and appearance of both CD4 and CD8+ T cells that express NKRs. (+info)Local and systemic delivery of a stable aspirin-triggered lipoxin prevents neutrophil recruitment in vivo. (7/1118)
Aspirin (ASA) triggers a switch in the biosynthesis of lipid mediators, inhibiting prostanoid production and initiating 15-epi-lipoxin generation through the acetylation of cyclooxygenase II. These aspirin-triggered lipoxins (ATL) may mediate some of ASA's beneficial actions and therefore are of interest in the search for novel antiinflammatories that could manifest fewer unwanted side effects. Here, we report that design modifications to native ATL structure prolong its biostability in vivo. In mouse whole blood, ATL analogs protected at carbon 15 [15(R/S)-methyl-lipoxin A4 (ATLa1)] and the omega end [15-epi-16-(para-fluoro)-phenoxy-LXA4 (ATLa2)] were recoverable to approximately 90 and 100% at 3 hr, respectively, compared with a approximately 40% loss of native lipoxin A4. ATLa2 retains bioactivity and, at levels as low as approximately 24 nmol/mouse, potently inhibited tumor necrosis factor-alpha-induced leukocyte recruitment into the dorsal air pouch. Inhibition was evident by either local intra-air pouch delivery (approximately 77% inhibition) or systemic delivery by intravenous injection (approximately 85% inhibition) and proved more potent than local delivery of ASA. Rank order for inhibiting polymorphonuclear leukocyte infiltration was: ATLa2 (10 micrograms, i.v.) approximately ATLa2 (10 micrograms, local) approximately dexamethasone (10 micrograms, local) >ASA (1.0 mg, local). Applied topically to mouse ear skin, ATLa2 also inhibited polymorphonuclear leukocyte infiltration induced by leukotriene B4 (approximately 78% inhibition) or phorbol ester (approximately 49% inhibition), which initiates endogenous chemokine production. These results indicate that this fluorinated analog of natural aspirin-triggered lipoxin A4 is bioavailable by either local or systemic delivery routes and is a more potent and precise inhibitor of neutrophil accumulation than is ASA. (+info)Histamine response and local cooling in the human skin: involvement of H1- and H2-receptors. (8/1118)
AIMS: Histamine may contribute locally to cutaneous blood flow control under normal and pathologic conditions. The objective of this study was to observe the influence of skin temperature on histamine vasodilation, and the roles of H1-and H2-receptors using novel noninvasive methods. METHODS: Eleven healthy subjects received, double-blind, single doses of the H1-receptor antagonist cetirizine (10 mg), cetirizine (10 mg) plus the H2-receptor antagonist cimetidine (400 mg), or placebo on separate occasions. Histamine was dosed cumulatively by iontophoresis to the forearm skin at 34 degrees C and 14 degrees C. Laser-Doppler flux (LDF) was measured at the same sites using customised probeholder/iontophoretic chambers with Peltier cooling elements. Finger mean arterial pressure (MAP) was measured and cutaneous vascular conductance calculated as LDF/MAP. RESULTS: Histamine vasodilation was reduced in cold skin. Cetirizine shifted the histamine dose-response at both temperatures: statistically significantly at 14 degrees C only. Combined H1- and H2-receptor antagonism shifted the response significantly at both temperatures. CONCLUSIONS: H1- and H2-receptors mediate histamine-induced skin vasodilation. The sensitivity of these receptors, particularly the H1- receptor, is attenuated at low skin temperature. Whether the reduced effect in cold skin represents specific receptor or postreceptor desensitization, or nonspecific attenuation of cutaneous vasodilation remains to be elucidated. (+info)Some common types of skin diseases include:
1. Acne: a condition characterized by oil clogged pores, pimples, and other blemishes on the skin.
2. Eczema: a chronic inflammatory skin condition that causes dry, itchy, and scaly patches on the skin.
3. Psoriasis: a chronic autoimmune skin condition characterized by red, scaly patches on the skin.
4. Dermatitis: a term used to describe inflammation of the skin, often caused by allergies or irritants.
5. Skin cancer: a type of cancer that affects the skin cells, often caused by exposure to UV radiation from the sun or tanning beds.
6. Melanoma: the most serious type of skin cancer, characterized by a mole that changes in size, shape, or color.
7. Vitiligo: a condition in which white patches develop on the skin due to the loss of pigment-producing cells.
8. Alopecia: a condition characterized by hair loss, often caused by autoimmune disorders or genetics.
9. Nail diseases: conditions that affect the nails, such as fungal infections, brittleness, and thickening.
10. Mucous membrane diseases: conditions that affect the mucous membranes, such as ulcers, inflammation, and cancer.
Skin diseases can be diagnosed through a combination of physical examination, medical history, and diagnostic tests such as biopsies or blood tests. Treatment options vary depending on the specific condition and may include topical creams or ointments, oral medications, light therapy, or surgery.
Preventive measures to reduce the risk of skin diseases include protecting the skin from UV radiation, using sunscreen, wearing protective clothing, and avoiding exposure to known allergens or irritants. Early detection and treatment can help prevent complications and improve outcomes for many skin conditions.
There are several types of skin neoplasms, including:
1. Basal cell carcinoma (BCC): This is the most common type of skin cancer, and it usually appears as a small, fleshy bump or a flat, scaly patch. BCC is highly treatable, but if left untreated, it can grow and invade surrounding tissue.
2. Squamous cell carcinoma (SCC): This type of skin cancer is less common than BCC but more aggressive. It typically appears as a firm, flat, or raised bump on sun-exposed areas. SCC can spread to other parts of the body if left untreated.
3. Melanoma: This is the most serious type of skin cancer, accounting for only 1% of all skin neoplasms but responsible for the majority of skin cancer deaths. Melanoma can appear as a new or changing mole, and it's essential to recognize the ABCDE signs (Asymmetry, Border irregularity, Color variation, Diameter >6mm, Evolving size, shape, or color) to detect it early.
4. Sebaceous gland carcinoma: This rare type of skin cancer originates in the oil-producing glands of the skin and can appear as a firm, painless nodule on the forehead, nose, or other oily areas.
5. Merkel cell carcinoma: This is a rare and aggressive skin cancer that typically appears as a firm, shiny bump on the skin. It's more common in older adults and those with a history of sun exposure.
6. Cutaneous lymphoma: This type of cancer affects the immune system and can appear as a rash, nodules, or tumors on the skin.
7. Kaposi sarcoma: This is a rare type of skin cancer that affects people with weakened immune systems, such as those with HIV/AIDS. It typically appears as a flat, red or purple lesion on the skin.
While skin cancers are generally curable when detected early, it's important to be aware of your skin and notice any changes or unusual spots, especially if you have a history of sun exposure or other risk factors. If you suspect anything suspicious, see a dermatologist for an evaluation and potential biopsy. Remember, prevention is key to avoiding the harmful effects of UV radiation and reducing your risk of developing skin cancer.
The exact cause of Raynaud disease is not fully understood, but it is believed to be related to an autoimmune disorder, in which the body's immune system mistakenly attacks healthy tissue. The condition can occur on its own or as a secondary symptom of another underlying medical condition such as scleroderma or rheumatoid arthritis.
Symptoms of Raynaud Disease:
1) Discoloration: Raynaud disease causes the affected areas to turn white or blue in response to cold temperatures or stress.
2) Pain: The constriction of blood vessels can cause pain in the affected areas.
3) Numbness or tingling: The lack of blood flow can cause numbness or tingling sensations in the fingers and toes.
4) Swelling: In severe cases, swelling may occur in the affected areas.
5) Burning sensation: Some people with Raynaud disease may experience a burning sensation in their hands and feet.
Diagnosis of Raynaud Disease:
1) Medical history: A doctor will ask about symptoms, medical history, and any triggers that may cause the condition.
2) Physical examination: The doctor will perform a physical examination to look for signs of discoloration or swelling in the affected areas.
3) Tests: Additional tests such as nailfold capillary microscopy, pulse volume recording and thermography may be ordered to confirm the diagnosis.
Treatment options for Raynaud Disease:
1) Medications: Drugs such as calcium channel blockers, alpha-blockers, and anticoagulants can help to relax blood vessels and improve blood flow.
2) Lifestyle changes: Avoiding triggers such as cold temperatures and taking steps to keep hands and feet warm can help manage the condition.
3) Alternative therapies: Some people with Raynaud disease may find relief with alternative therapies such as acupuncture or biofeedback.
It is important to note that in some cases, Raynaud disease can be a symptom of an underlying autoimmune disorder, such as lupus or scleroderma. If you suspect you have Raynaud disease, it is essential to seek medical attention to rule out any other conditions.
Types of Skin Ulcers:
1. Pressure ulcers (bedsores): These occur when pressure is applied to a specific area of the skin for a long time, causing the skin to break down. They are more common in people who are bedridden or have mobility issues.
2. Diabetic foot ulcers: These are caused by nerve damage and poor circulation in people with diabetes, which can lead to unnoticed injuries or infections that do not heal properly.
3. Venous ulcers: These occur when the veins have difficulty returning blood to the heart, causing pressure to build up in the legs and feet. This pressure can cause skin breakdown and ulceration.
4. Arterial ulcers: These are caused by poor circulation due to blockages or narrowing of the arteries, which can lead to a lack of oxygen and nutrients to the skin.
5. Traumatic ulcers: These are caused by injuries or surgery and can be shallow or deep.
Symptoms of Skin Ulcers:
1. Pain
2. Redness around the wound
3. Swelling
4. Discharge or pus
5. A foul odor
6. Increased pain when touched or pressure is applied
7. Thick, yellowish discharge
8. Skin that feels cool to the touch
9. Redness that spreads beyond the wound margins
10. Fever and chills
Treatment for Skin Ulcers:
1. Debridement: Removing dead tissue and bacteria from the wound to promote healing.
2. Dressing changes: Applying a dressing that absorbs moisture, protects the wound, and promotes healing.
3. Infection control: Administering antibiotics to treat infections and prevent further complications.
4. Pain management: Managing pain with medication or other interventions.
5. Offloading pressure: Reducing pressure on the wound using specialized mattresses, seat cushions, or orthotics.
6. Wound cleansing: Cleaning the wound with saline solution or antimicrobial agents to remove bacteria and promote healing.
7. Nutritional support: Providing adequate nutrition to promote wound healing.
8. Monitoring for signs of infection: Checking for signs of infection, such as increased redness, swelling, or drainage, and addressing them promptly.
9. Addressing underlying causes: Managing underlying conditions, such as diabetes or poor circulation, to promote wound healing.
10. Surgical intervention: In some cases, surgery may be necessary to promote wound healing or repair damaged tissue.
Prevention of pressure sores is always preferable to treatment, and this can be achieved by taking steps such as:
1. Turning and repositioning regularly: Changing positions regularly, at least every two hours, to redistribute pressure.
2. Using pressure-relieving support surfaces: Using mattresses or cushions that reduce pressure on the skin.
3. Keeping the skin clean and dry: Ensuring the skin is clean and dry, especially after incontinence or sweating.
4. Monitoring nutrition and hydration: Ensuring adequate nutrition and hydration to support healing.
5. Managing underlying conditions: Managing conditions such as diabetes, poor circulation, or immobility to reduce the risk of pressure sores.
6. Using barrier creams or films: Applying barrier creams or films to protect the skin from moisture and friction.
7. Providing adequate cushioning: Using cushions or pillows that provide adequate support and reduce pressure on the skin.
8. Encouraging mobility: Encouraging regular movement and exercise to improve circulation and reduce immobility.
9. Monitoring for signs of pressure sores: Regularly checking for signs of pressure sores, such as redness, swelling, or pain.
10. Seeking medical advice if necessary: Seeking medical advice if pressure sores are suspected or if there are any concerns about their prevention or treatment.
1. Impetigo: A highly contagious infection that causes red sores on the face, arms, and legs. It is most commonly seen in children and is usually treated with antibiotics.
2. Cellulitis: A bacterial infection of the skin and underlying tissue that can cause swelling, redness, and warmth. It is often caused by Streptococcus or Staphylococcus bacteria and may require hospitalization for treatment.
3. MRSA (Methicillin-resistant Staphylococcus aureus): A type of staph infection that is resistant to many antibiotics and can cause severe skin and soft tissue infections. It is often seen in hospitals and healthcare settings and can be spread through contact with an infected person or contaminated surfaces.
4. Erysipelas: A bacterial infection that causes red, raised borders on the skin, often on the face, legs, or arms. It is caused by Streptococcus bacteria and may require antibiotics to treat.
5. Folliculitis: An infection of the hair follicles that can cause redness, swelling, and pus-filled bumps. It is often caused by Staphylococcus bacteria and may be treated with antibiotics or topical creams.
6. Boils: A type of abscess that forms when a hair follicle or oil gland becomes infected. They can be caused by either Staphylococcus or Streptococcus bacteria and may require draining and antibiotics to treat.
7. Carbuncles: A type of boil that is larger and more severe, often requiring surgical drainage and antibiotics to treat.
8. Erythrasma: A mild infection that causes small, red patches on the skin. It is caused by Corynebacterium bacteria and may be treated with antibiotics or topical creams.
9. Cellulitis: An infection of the deeper layers of skin and subcutaneous tissue that can cause swelling, redness, and warmth in the affected area. It is often caused by Staphylococcus bacteria and may require antibiotics to treat.
10. Impetigo: A highly contagious infection that causes red sores or blisters on the skin, often around the nose, mouth, or limbs. It is caused by Staphylococcus or Streptococcus bacteria and may be treated with antibiotics or topical creams.
These are just a few examples of common skin infections and there are many more types that can occur. If you suspect you or someone else has a skin infection, it's important to seek medical attention as soon as possible for proper diagnosis and treatment.
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List of MeSH codes
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Support surface
GSR
Sense
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Orienting response
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Saliva testing
Perspiration
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Immune tolerance
Neuroeconomics
Electrodermal activity
Entoptic phenomenon
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Normal distribution
Automated Pain Recognition
Affect labeling
Voodoo death
Non-invasive micro-test technology
Amorphosynthesis
Neodymium
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Senescence
Conservation biology
Physiologically based pharmacokinetic modelling
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Timeline of United States inventions (1890-1945)
Catch bond
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Transgenerational trauma
Emotion perception
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Cognitive aspects underlying pain and neuro-physiological responses
Normal physiological2
- The studies on the aryl hydrocarbon receptor (AHR) discussed here provide a perfect example of how years of basic toxicological research on a molecule, whose normal physiological function remained a mystery for so long, has now yielded a treasure trove of actionable information on the development of targeted therapeutics. (nih.gov)
- Sweating is a normal physiological phenomenon that the body needs to maintain internal body temperature within normal ratios as well as regulating body level of fluids and electrolytes. (tophealthblog.com)
Psychological8
- Furthermore, the physiological and psychological effects of higher frequency sound and noise comprise an enormous volume of literature and have not been reviewed at this time. (nih.gov)
- Rather, the document focuses on identifying and describing the available literature regarding reported physiological and/or psychological effects of infrasound. (nih.gov)
- A state of comfort can only be achieved when the most complex interactions between a range of physiological, psychological and physical factors have taken place in a satisfactory manner. (scialert.net)
- What are the psychological implications of the natural variation in skin pigmentation that renders the blush more or less noticeable? (raycrozier.com)
- It includes discussions of psychological variables related to psychic phenomena, for instance with regard to personality, and of psychological concepts that are used to make sense of ESP and other phenomena. (spr.ac.uk)
- Psychic phenomena manifest dynamic aspects, and personality and cognitive variables, suggesting they are part of normal psychological processes. (spr.ac.uk)
- The power of the placebo effect is considered to be a psychological phenomenon. (medicalnewstoday.com)
- Hadjistavropoulos and Craig do a little better by calling nociception "physiological" and pain "psychological. (drivingwithnohands.com)
Processes2
- It's a key constituent of all physiological processes in the body. (sscatoz.com)
- What is its physiological basis and how does this differ from the processes underlying, for example, anger or indignation? (raycrozier.com)
Humans1
- Though some animals do have the physiological ability to produce tears, humans are the only creatures whose tears can be triggered emotionally . (everydaysociologyblog.com)
Measurable1
- Placebos have been shown to produce measurable, physiological changes, such as an increase in heart rate or blood pressure . (medicalnewstoday.com)
Temperature6
- Skin to skin care has been practised in primitive and high technology cultures for body temperature preservation in neonates. (nih.gov)
- Regional skin temperature and heat flow was measured in moderately hypothermic term neonates to quantitate the heat transfer occurring during one hour of skin to skin care. (nih.gov)
- Nine healthy newborns with a mean rectal temperature of 36.3 degrees C were placed skin to skin on their mothers' chests. (nih.gov)
- The mean (SD) rectal temperature increased by 0.7 (0.4) degrees C to 37.0 degrees C. The heat loss was high (70 Wm-2) from the unprotected skin of the head to the surrounding air. (nih.gov)
- Decrease of skin blood flow in fingers to 6.3 milligrams per 100 grams of tissue per minute, an increase in peripheral resistance, and a drop of skin temperature were observed. (cdc.gov)
- Whereas signs of fear such as pallor, trembling and 'butterflies in the stomach' seem understandable in terms of the body's preparation for action the utility of reddening of the face and increased skin temperature is not so evident. (raycrozier.com)
Metabolic1
- Researchers thought that by providing gamma-linolenic acid by ingestion for organisms that could not produce it themselves, the formation of PG1 would be restored and, at the same time, the physiological and metabolic disorders accompanying this deficiency would be cured. (holistica.fr)
Subjective1
- The textbook uses the nouns "transduction, transmission, and modulation," lumps these together under a new noun, "nociception," and claims that this is the "objective" phenomenon, something that's happening in our body on which we construct the "subjective" phenomenon, the more amorphous thing, we call "pain. (drivingwithnohands.com)
Radiation1
- Radiation effects on the skin. (nih.gov)
Pigmentation1
- It includes the pigmentation of the skin. (bvsalud.org)
Inflammatory skin1
- These compounds contain anti-inflammatory properties and might guard against inflammatory skin conditions like acne and psoriasis. (sscatoz.com)
Acne2
- Acne is an epidemic skin disease of industrialized countries, reaching prevalence rates of over 85 percent of teenagers. (nutritionfacts.org)
- So, acne is not some "physiological" phenomenon of puberty, but may represent "a visible risk indicator pointing to aberrant nutrient signaling promoting chronic epidemic diseases of civilization," according to a group of German researchers (See Saving Lives By Treating Acne With Diet ). (nutritionfacts.org)
Tissues1
- Many nerve receptors lie close together in the skin and underlying tissues. (drivingwithnohands.com)
Unconscious2
- anti-synechistic thinkers wind themselves up in a factitious snarl by falsifying the phenomena in representing consciousness to be, as it were, a skin, a separate tissue, overlying an unconscious region of the occult nature, mind, soul, or physiological basis. (commens.org)
- A particular phenomenon that Rhine and Pratt associated with the unconscious is the tendency of certain subjects in ESP tests to obtain results that are significantly below chance, that is, to score lower than the average in a way that is just as anomalous as if they had scored above it. (spr.ac.uk)
Basis1
- Several theoretical frameworks have been proposed to explain the physiological basis of pain, although none yet completely accounts for all aspects of pain perception. (slideshare.net)
Effects1
- Diets loaded with MUFAs can fight premature skin aging due to their anti-inflammatory effects. (sscatoz.com)
Diseases4
Physical1
- Sound is a complex physical phenomenon and no attempt is made here to describe in detail acoustic principles or methods for the measurement of sound. (nih.gov)
Body1
- The functions of the skin in the human and animal body. (bvsalud.org)
Prevention1
- In a time where "translational" science has become a mantra in the biomedical field, it is reassuring when years of research into a biological phenomenon suddenly points towards novel prevention or therapeutic approaches to disease, thereby demonstrating once again that basic science and translational science are intimately linked. (nih.gov)
Resistance1
- In addition to this, basic properties expected from socks during usage are resistance against abrasion, elasticity, constant post-washing dimensions, thermo-physiological properties and physiological comfort. (scialert.net)
Pain1
- The capacity to burst into tears due to emotional stress or pain shows us how tears are a social phenomenon. (everydaysociologyblog.com)
Function1
- Disorders of thermoregulating function of skin circulation seem to be the cause of vibration disease. (cdc.gov)
Complex1
- Examining such a complex and fascinating phenomenon has moved the boundaries of linguistic research. (au.dk)
Properties1
- It is known that fiber type, yarn properties, fabric structure, finishing treatments and clothing conditions are the main factors affecting thermo-physiological comfort ( Li, 2001 ). (scialert.net)
Heat3
- and heat was initially gained from areas in contact with the mother's skin. (nih.gov)
- The total dry heat loss during skin to skin care corresponded to heat loss during incubator care at 32-32.5 degrees C. The reduced heat loss, and to a minor extent, the initial heat flux from the mothers allowed heat to be conserved, leading to rewarming. (nih.gov)
- Providing optimum heat, damp and air passing are expected from socks through physiological comfort. (scialert.net)
Increase2
- This confirmed an observed increase in skin arterioles tonus. (cdc.gov)
- Blood flow to the skin also tends to increase during sleep. (sscatoz.com)
Time2
- It is the time when biting winds gnaw on our skin and whip chilly, wintry air into our eyelashes. (everydaysociologyblog.com)
- C ONSIDERING the length of time that so-called "animal magnetism," "mesmerism," or "electro-biology," has been before the world, it is a matter of surprise that so inviting a field of physiological inquiry should have been so long allowed to lie fallow. (wikisource.org)
Cells1
- Free radicals are associated with oxidative damage to cells, a phenomenon linked to high risks of disease. (sscatoz.com)
Warm1
- And with thermo-physiological comfort, perceiving the fabric comfort on the skin and feelings providing sensations of warm, cold, wet and touch positively are understood ( Ozdil, 2008 ). (scialert.net)