Magnetic Resonance Imaging
Ventral Tegmental Area
Analysis of Variance
Dopamine Uptake Inhibitors
Substance Withdrawal Syndrome
Image Processing, Computer-Assisted
Task Performance and Analysis
Receptors, Dopamine D2
Medial Forebrain Bundle
Cladistic association analysis of Y chromosome effects on alcohol dependence and related personality traits. (1/3403)Association between Y chromosome haplotype variation and alcohol dependence and related personality traits was investigated in a large sample of psychiatrically diagnosed Finnish males. Haplotypes were constructed for 359 individuals using alleles at eight loci (seven microsatellite loci and a nucleotide substitution in the DYZ3 alphoid satellite locus). A cladogram linking the 102 observed haplotype configurations was constructed by using parsimony with a single-step mutation model. Then, a series of contingency tables nested according to the cladogram hierarchy were used to test for association between Y haplotype and alcohol dependence. Finally, using only alcohol-dependent subjects, we tested for association between Y haplotype and personality variables postulated to define subtypes of alcoholism-antisocial personality disorder, novelty seeking, harm avoidance, and reward dependence. Significant association with alcohol dependence was observed at three Y haplotype clades, with significance levels of P = 0.002, P = 0.020, and P = 0.010. Within alcohol-dependent subjects, no relationship was revealed between Y haplotype and antisocial personality disorder, novelty seeking, harm avoidance, or reward dependence. These results demonstrate, by using a fully objective association design, that differences among Y chromosomes contribute to variation in vulnerability to alcohol dependence. However, they do not demonstrate an association between Y haplotype and the personality variables thought to underlie the subtypes of alcoholism. (+info)
An analysis of choice making in the assessment of young children with severe behavior problems. (2/3403)We examined how positive and negative reinforcement influenced time allocation, occurrence of problem behavior, and completion of parent instructions during a concurrent choice assessment with 2 preschool-aged children who displayed severe problem behavior in their homes. The children were given a series of concurrent choice options that varied availability of parent attention, access to preferred toys, and presentation of parent instructions. The results showed that both children consistently allocated their time to choice areas that included parent attention when no instructions were presented. When parent attention choice areas included the presentation of instructions, the children displayed differential patterns of behavior that appeared to be influenced by the presence or absence of preferred toys. The results extended previous applications of reinforcer assessment procedures by analyzing the relative influence of both positive and negative reinforcement within a concurrent-operants paradigm. (+info)
Delay or probability discounting in a model of impulsive behavior: effect of alcohol. (3/3403)Little is known about the acute effects of drugs of abuse on impulsivity and self-control. In this study, impulsivity was assessed in humans using a computer task that measured delay and probability discounting. Discounting describes how much the value of a reward (or punisher) is decreased when its occurrence is either delayed or uncertain. Twenty-four healthy adult volunteers ingested a moderate dose of ethanol (0.5 or 0.8 g/kg ethanol: n = 12 at each dose) or placebo before completing the discounting task. In the task the participants were given a series of choices between a small, immediate, certain amount of money and $10 that was either delayed (0, 2, 30, 180, or 365 days) or probabilistic (i.e., certainty of receipt was 1.0, .9, .75, .5, or .25). The point at which each individual was indifferent between the smaller immediate or certain reward and the $10 delayed or probabilistic reward was identified using an adjusting-amount procedure. The results indicated that (a) delay and probability discounting were well described by a hyperbolic function; (b) delay and probability discounting were positively correlated within subjects; (c) delay and probability discounting were moderately correlated with personality measures of impulsivity; and (d) alcohol had no effect on discounting. (+info)
Sodium depletion and aldosterone decrease dopamine transporter activity in nucleus accumbens but not striatum. (4/3403)Motivated behaviors, including sodium (Na) appetite, are correlated with increased dopamine (DA) transmission in the nucleus accumbens (NAc). DA transporter (DAT) modulation affects DA transmission and may play a role in motivated behaviors. In vivo Na depletion, which reliably induces Na appetite, was correlated with robust decreases in DA uptake via the DAT in the rat NAc with rotating disk electrode voltammetry [1,277 +/- 162 vs. 575 +/- 89 pmol. s-1. g-1; Vmax of transport for control vs. Na-depleted tissue]. Plasma aldosterone (Aldo) levels increase after in vivo Na depletion and contribute to Na appetite. Decreased DAT activity in the NAc was observed after in vitro Aldo treatment (428 +/- 28 vs. 300 +/- 25 pmol. s-1. g-1). Neither treatment affected DAT activity in the striatum. These results suggest that a direct action of Aldo is one possible mechanism by which Na depletion induces a reduction in DAT activity in the NAc. Reduced DAT activity may play a role in generating increased NAc DA transmission during Na appetite, which may underlie the motivating properties of Na for the Na-depleted rat. (+info)
Dopamine fluctuations in the nucleus accumbens during maintenance, extinction, and reinstatement of intravenous D-amphetamine self-administration. (5/3403)Moment-to-moment fluctuations of nucleus accumbens dopamine (DA) were determined in rats self-administering or passively receiving "yoked" intravenous infusions of D-amphetamine. The initial lever presses of each session caused elevations in DA concentration, usually to an initial peak that was not maintained throughout the rest of the session. As the initial ("loading") injections were metabolized, DA levels dropped toward baseline but were sustained at elevated plateaus by subsequent lever pressing that was spaced throughout the remainder of the 3 hr sessions. During this period, DA levels fluctuated phasically, time-locked to the cycle of periodic lever pressing. Consistent with the known pharmacological actions and dynamics of amphetamine, peak DA elevations were seen approximately 10-15 min after each injection, and the mean DA level was at a low point in the phasic cycle at the time of each new lever press. During extinction periods when saline was substituted for amphetamine, DA levels dropped steadily toward baseline levels despite a dramatic increase in (now-unrewarded) lever pressing. Noncontingent injections during extinction reinstated lever-pressing behavior and increased nucleus accumbens DA concentrations. These data are consistent with the hypothesis that under the conditions of this experiment-during periods of amphetamine intoxication in well-trained animals-the timing of amphetamine self-administration comes primarily under the control of extracellular DA concentrations. The probability of lever pressing during the maintenance phase is highest when DA concentrations fall near a characteristic trigger point, a trigger point that is significantly elevated above baseline, and falls as DA concentrations fall below or increase above that trigger point. (+info)
Enhancement of locomotor activity and conditioned reward to cocaine by brain-derived neurotrophic factor. (6/3403)The mesolimbic dopamine (DA) system has been implicated in drug reward, locomotor sensitization, and responding for reward-related stimuli [termed conditioned reinforcers (CR)]. Here, we investigated the effect of brain-derived neurotrophic factor (BDNF), which enhances the survival and function of dopaminergic neurons, on stimulant-induced locomotor sensitization and responding for CR. In experiment 1, BDNF was infused into the nucleus accumbens (NAc) or ventral tegmental area over 2 weeks via chronically implanted minipumps (1-2.5 microgram/d), and the psychomotor stimulant effects of cocaine (5-15 mg/kg, i.p.) were studied. We found that BDNF enhanced the initial stimulant effects of cocaine and seemed to facilitate the development of sensitization to repeated cocaine doses. In experiment 2, we studied the effects of intra-NAc BDNF infusions on responding for CR. BDNF-treated rats showed twice as many CR responses compared with controls when saline was first administered. BDNF enhanced responding on the CR lever more than four times that seen in control animals after a cocaine injection (10 mg/kg, i.p.). The enhanced response to cocaine in BDNF-treated animals persisted for more than a month after the BDNF infusions had stopped, indicating long-lasting changes in the mesolimbic DA system caused by BDNF administration. In experiment 3, we examined locomotor sensitization to cocaine in heterozygous BDNF knock-out mice and found that the development of sensitization was delayed compared with wild-type littermates. These results demonstrate the profound effects of BDNF on the enhancement of both cocaine-induced locomotion and facilitation of CR and suggest a possible role for BDNF in long-term adaptations of the brain to cocaine. (+info)
High effort, low reward, and cardiovascular risk factors in employed Swedish men and women: baseline results from the WOLF Study. (7/3403)STUDY OBJECTIVE: To examine associations between measures of work stress (that is, the combination of high effort and low reward) and cardiovascular risk factors. DESIGN: Cross sectional first screening of a prospective cohort study. SETTING AND PARTICIPANTS: The study was conducted among 5720 healthy employed men and women living in the greater Stockholm area aged 19-70 years. All analyses were restricted to subjects with complete data (n = 4958). The investigation of associations between indicators of effort-reward imbalance and cardiovascular risk factors was restricted to the age group 30-55 years (n = 3427). MAIN RESULTS: Subjects reporting high effort and low reward at work had a higher prevalence of well known risk factors for coronary heart disease. After adjustment for relevant confounders, associations between a measure of extrinsic effort and reward (the effort-reward ratio) and hypertension (multivariate prevalence odds ratio (POR) 1.62-1.68), increased total cholesterol (upper tertile 220 mg/dl)(POR = 1.24) and the total cholesterol/high density lipoprotein(HDL)-cholesterol ratio (upper tertile 4.61)(POR 1.26-1.30) were found among men. Among women a measure of high intrinsic effort (immersion) was related to increased low density lipoprotein (LDL)-cholesterol (upper tertile 130 mg/dl)(POR 1.37-1.39). Analyses of variance showed increasing mean values of LDL cholesterol with an increasing degree of the effort-reward ratio among men and increased LDL-cholesterol among women with high levels of intrinsic effort (upper tertile of immersion). CONCLUSIONS: Findings lend support to the hypothesis that effort-reward imbalance represents a specific constellation of stressful experience at work related to cardiovascular risk. The relation was not explained by relevant confounders (for example, lack of physical exercise, body weight, cigarette smoking). (+info)
Muscimol-induced inactivation of monkey frontal eye field: effects on visually and memory-guided saccades. (8/3403)Muscimol-induced inactivation of the monkey frontal eye field: effects on visually and memory-guided saccades. Although neurophysiological, anatomic, and imaging evidence suggest that the frontal eye field (FEF) participates in the generation of eye movements, chronic lesions of the FEF in both humans and monkeys appear to cause only minor deficits in visually guided saccade generation. Stronger effects are observed when subjects are tested in tasks with more cognitive requirements. We tested oculomotor function after acutely inactivating regions of the FEF to minimize the effects of plasticity and reallocation of function after the loss of the FEF and gain more insight into the FEF contribution to the guidance of eye movements in the intact brain. Inactivation was induced by microinjecting muscimol directly into physiologically defined sites in the FEF of three monkeys. FEF inactivation severely impaired the monkeys' performance of both visually guided and memory-guided saccades. The monkeys initiated fewer saccades to the retinotopic representation of the inactivated FEF site than to any other location in the visual field. The saccades that were initiated had longer latencies, slower velocities, and larger targeting errors than controls. These effects were present both for visually guided and for memory-guided saccades, although the memory-guided saccades were more disrupted. Initially, the effects were restricted spatially, concentrating around the retinotopic representation at the center of the inactivated site, but, during the course of several hours, these effects spread to flanking representations. Predictability of target location and motivation of the monkey also affected saccadic performance. For memory-guided saccades, increases in the time during which the monkey had to remember the spatial location of a target resulted in further decreases in the accuracy of the saccades and in smaller peak velocities, suggesting a progressive loss of the capacity to maintain a representation of target location in relation to the fovea after FEF inactivation. In addition, the monkeys frequently made premature saccades to targets in the hemifield ipsilateral to the injection site when performing the memory task, indicating a deficit in the control of fixation that could be a consequence of an imbalance between ipsilateral and contralateral FEF activity after the injection. There was also a progressive loss of fixation accuracy, and the monkeys tended to restrict spontaneous visual scanning to the ipsilateral hemifield. These results emphasize the strong role of the FEF in the intact monkey in the generation of all voluntary saccadic eye movements, as well as in the control of fixation. (+info)
Gambling can also be considered a behavioral addiction, as some individuals may become so consumed by the activity that they neglect other aspects of their lives, experience financial problems, and exhibit other signs of addiction. In this context, gambling is often classified as an impulse control disorder or a substance use disorder.
In the medical field, gambling can have various effects on an individual's physical and mental health, such as:
1. Financial problems: Gambling can lead to significant financial losses, which can cause stress, anxiety, and depression.
2. Sleep disturbances: Engaging in gambling activities at night or experiencing the excitement of winning can disrupt sleep patterns and lead to insomnia or other sleep disorders.
3. Substance abuse: Gambling can sometimes be accompanied by substance abuse, as individuals may turn to drugs or alcohol to cope with their gambling problems or to enhance their gambling experience.
4. Mood disorders: Gambling can contribute to the development of mood disorders such as depression, anxiety, and bipolar disorder.
5. Suicidal ideation: In extreme cases, individuals struggling with gambling addiction may experience suicidal thoughts or attempts.
6. Social problems: Gambling can strain relationships with family and friends, leading to social isolation and loneliness.
7. Physical health problems: Chronic stress and anxiety associated with gambling can contribute to various physical health problems, such as hypertension, cardiovascular disease, and musculoskeletal problems.
8. Cognitive impairment: Compulsive gambling can affect cognitive functioning, including attention, memory, and decision-making abilities.
9. Family dynamics: Gambling can have a significant impact on family dynamics, leading to conflicts, divorce, and financial hardship.
10. Financial consequences: Gambling can lead to significant financial problems, including debt, bankruptcy, and even criminal activity.
It's important to note that not all individuals who experience these problems will develop a gambling disorder, and that other factors such as genetics, family history, and environmental factors can contribute to the development of gambling addiction.
Anhedonia can manifest in different ways, depending on the individual and their specific condition. Some common examples include:
* Loss of interest in activities that were once enjoyed, such as hobbies or socializing
* Difficulty experiencing pleasure from activities that are normally enjoyable, such as eating or sexual activity
* Feeling emotionally flat or numb, even in response to positive events or experiences
* Difficulty finding joy or happiness in life, even in response to positive events or experiences.
Anhedonia can be caused by a wide range of factors, including:
* Depression and other mood disorders
* Schizophrenia and other psychotic disorders
* Neurological disorders such as Parkinson's disease, Huntington's disease, and multiple sclerosis
* Chronic pain and other conditions that can affect the brain's reward system
* Substance abuse and addiction
* Sleep disorders
* Nutritional deficiencies, such as a lack of vitamin B12 or iron.
There are several ways to diagnose anhedonia, including:
* Clinical interview: A healthcare professional will ask questions about the patient's symptoms and medical history to determine if they are experiencing anhedonia.
* Physical examination: The healthcare professional may also perform a physical examination to rule out any underlying medical conditions that could be causing the anhedonia.
* Psychological assessments: The healthcare professional may use standardized tests to assess the patient's mood and emotional state, such as the Hamilton Rating Scale for Depression or the Beck Depression Inventory.
There are several treatment options for anhedonia, depending on the underlying cause. These may include:
* Medications: Antidepressants, antipsychotics, and mood stabilizers can be effective in treating anhedonia caused by depression and other mental health conditions.
* Psychotherapy: Talk therapy, such as cognitive-behavioral therapy (CBT), can help patients identify and change negative thought patterns and behaviors that contribute to anhedonia.
* Lifestyle changes: Making healthy lifestyle changes, such as regular exercise, getting enough sleep, and eating a balanced diet, can help improve mood and reduce anhedonia.
It is important to seek medical attention if you or someone you know is experiencing symptoms of anhedonia, as early diagnosis and treatment can improve the chances of successful treatment.
* Muscle and bone pain
* Nausea and vomiting
* Seizures (in severe cases)
The specific symptoms of substance withdrawal syndrome can vary depending on the substance being withdrawn from, but some common symptoms include:
* Alcohol: tremors, anxiety, insomnia, nausea and vomiting, headaches, and seizures
* Opioids: withdrawal symptoms can include anxiety, muscle aches, sweating, nausea and vomiting, diarrhea, and depression
* Benzodiazepines: withdrawal symptoms can include anxiety, insomnia, tremors, and seizures
The diagnosis of substance withdrawal syndrome is typically made based on the patient's history of substance use and the presence of withdrawal symptoms. A healthcare provider may also order laboratory tests to rule out other conditions that may be causing the symptoms. Treatment for substance withdrawal syndrome usually involves supportive care, such as rest, hydration, and pain management, as well as medication to manage withdrawal symptoms. In some cases, medical professionals may also recommend a gradual tapering of the substance over a period of time to minimize withdrawal symptoms.
It is important for individuals who are experiencing withdrawal symptoms to seek medical attention as soon as possible, as untreated withdrawal can lead to serious complications, such as seizures and dehydration. With appropriate treatment, most individuals with substance withdrawal syndrome can recover fully and successfully overcome their addiction.
1. Cocaine dependence: This is a condition in which an individual becomes psychologically and physiologically dependent on cocaine, and experiences withdrawal symptoms when they stop using the drug.
2. Cocaine intoxication: This is a state of altered consciousness that can occur when an individual takes too much cocaine, and can cause symptoms such as agitation, confusion, and hallucinations.
3. Cocaine-induced psychosis: This is a condition in which an individual experiences a break from reality, characterized by delusions, hallucinations, and disorganized thinking.
4. Cocaine-associated cardiovascular problems: Cocaine use can increase heart rate and blood pressure, and can cause damage to the heart and blood vessels.
5. Cocaine-associated respiratory problems: Cocaine use can constrict the airways and make breathing more difficult, which can lead to respiratory failure.
6. Cocaine-associated neurological problems: Cocaine use can cause nerve damage and seizures, particularly in individuals who use the drug frequently or in large quantities.
7. Cocaine withdrawal syndrome: This is a set of symptoms that can occur when an individual stops using cocaine, including depression, anxiety, and fatigue.
8. Cocaine-related anxiety disorders: Cocaine use can exacerbate anxiety disorders such as generalized anxiety disorder, panic disorder, and social anxiety disorder.
9. Cocaine-related mood disorders: Cocaine use can also exacerbate mood disorders such as depression and bipolar disorder.
10. Cocaine-related cognitive impairment: Chronic cocaine use can impair cognitive function, particularly in areas such as attention, memory, and decision-making.
It is important to note that the effects of cocaine can vary depending on the individual, the dose and frequency of use, and other factors such as the method of administration and any underlying medical conditions. If you or someone you know is struggling with cocaine addiction, it is important to seek professional help as soon as possible.
Risc vs. Reward
Reward (Shelltown, Maryland)
The $1,000,000 Reward
The Great Reward
Some Great Reward
Markov reward model
Reward Work Act
Kroger Community Rewards - Kroger
Promotion & Reward
MediaPost Publications - Tag: rewards
Talent and rewards
Reward System - Boomkat
Investors get little reward from complexity
French mystery rock: Village offers reward for deciphering inscription | CNN
Mysearch Global Rewards on Behance
Rush Hour Rewards - Google Nest 說明
NIMH » Reward Anticipation
Lucid Stock: A High-Risk, High-Reward EV Play
Reward, Employment Tax, Share Plans, HR & Payroll Advisory | Deloitte
Notable & Rewarding Family Caregiver Everyday Snapshot
How to Earn Hilton Reward Points For Taking Lyft Rides
Google To Reward Fixes For Open Source Software
Discover Marriott Bonvoy | Join The Best Hotel Rewards Program
The Ultimate Staff Reward | QSR magazine
Capella Progress Reward-$2,500 Master's Scholarship
Take a Vacation on Your Credit Card Rewards
GeForce Rewards & Giveaways | NVIDIA
Oakland police announce $10,000 reward for info in woman's slaying
Pay, Reward and Benefits | People and Culture | Queen's University Belfast
Maryland School District Rewards Teachers for Raising Test Scores
- We help you design cost-effective, tax-efficient, and compliant reward and payroll programs that align with talent strategies and diverse workforce needs. (deloitte.com)
- Together, we can create local and global reward programs that can benefit both your business and your employees-aligning them with talent strategies and your diverse workforce needs. (deloitte.com)
- We help you communicate your global incentive plan, equity, employee reward and employment tax plan in a way that can keep employees engaged using tailored creative communication strategies. (deloitte.com)
- The search for better therapeutic strategies for drug addiction raises the challenge to diminish motivation for drug without decreasing that for natural rewards. (nih.gov)
- Our results suggest distinct and dynamic neural population codes for natural reward and drug reward seeking in the mPFC and pave the way for future efforts in targeting specific neural codes for drug reward seeking as novel therapeutic strategies for drug addiction. (nih.gov)
- Application deadlines are coming up in the next few weeks to apply for several of NIH's High-Risk, High-Reward Research (HRHR) funding opportunities. (nih.gov)
- The researchers believe that reward (or anticipation of reward) reinforces neural circuits between reward and visual areas of the brain, and these circuits are then more likely to reactivate during sleep to facilitate task learning. (nih.gov)
- The ultimate goal of this study is to develop an integrative, data-driven model to examine how patterns of brain activation across functional domains give rise to distinct mechanisms underlying resilience, and how these neural mechanisms interrelate with behavioral (e.g., emotion regulation, reward responses, social cognition) and psychosocial (e.g., coping self-efficacy, positive emotions, social connectedness) factors implicated in resilience. (cdc.gov)
- While the medial prefrontal cortex (mPFC) is important for reward seeking, how prefrontal neural activities code reward seeking remains unknown. (nih.gov)
- Researchers have identified connections between neurons in brain systems associated with reward, stress, and emotion. (nih.gov)
- To map the brain circuitry between the BNST and the hypothalamus, Dr. Giardino and his colleagues exposed mice to rewarding and aversive stimuli, and then visualized and manipulated the activity of neurons using fiber optic techniques. (nih.gov)
- In the reward circuit, dopamine neurons release the neurotransmitter dopamine. (nih.gov)
- Dopamine surges in response to natural rewards help the brain learn and adapt to a complex world. (nih.gov)
- One of the ways in which we do this is through our Kroger Community Rewards program. (kroger.com)
- A digital account is needed to participate in Kroger Community Rewards. (kroger.com)
- Selecting the organization that you wish to support is as simple as updating the Kroger Community Rewards selection on your digital account. (kroger.com)
- Your selected organization will also display in the Kroger Community Rewards section of your account. (kroger.com)
- Kroger donates annually to participating organizations based on your percentage of spending as it relates to the total spending associated with all participating Kroger Community Rewards organizations. (kroger.com)
- Any seasoned brand, operator, or human-resources expert will tell you that rewarding your employees can bring about a host of positive results, from improved employee morale to better customer service. (qsrmagazine.com)
- Rewarding Adherence Program (RAP) is an individual-level intervention delivered to HIV clinic patients in Kampala, Uganda, who are antiretroviral therapy experienced. (cdc.gov)
- To complement its existing Vulnerability Reward Program for its Web applications and Chrome browser, the company has introduced Patch Rewards , a program that goes beyond flaw finding to flaw fixing: It will pay hackers for code contributions that get applied to certain open-source projects. (darkreading.com)
- High-Tech Bridge subsequently published a blog post to shame Yahoo and the ploy worked: Yahoo revised its bug bounty program and now offers rewards of $150 to $15,000 . (darkreading.com)
- But their inventory is limited and it takes time to accumulate miles -- especially when you don't travel often -- so it may be a good idea to transfer your credit card rewards to a partnering airline rewards program to snag a deal on a plane ticket. (businessinsider.com)
- The National Institutes of Health awarded 106 grants to support highly innovative and broadly impactful biomedical or behavioral research by exceptionally creative scientists through the Common Fund's High-Risk, High-Reward Research program. (nih.gov)
- The High-Risk, High-Reward Research program catalyzes scientific discovery by supporting highly innovative research proposals that, due to their inherent risk, may struggle in the traditional peer-review process despite their transformative potential. (nih.gov)
- The High-Risk, High-Reward Research Program is part of the NIH Common Fund , which oversees programs that pursue major opportunities and gaps throughout the research enterprise that are of great importance to NIH and require collaboration across the agency to succeed. (nih.gov)
- As part of the NIH's continual monitoring efforts, the NIH Director convened a Working Group of the Advisory Committee to the Director in Spring 2018 to examine the effectiveness of the Common Fund's High-Risk High-Reward Research Program and the diversity of its applicants and awardees. (nih.gov)
- The Working Group found that the High-Risk High-Reward Research Program overall is successful in supporting unusually innovative and impactful research. (nih.gov)
- The High-Risk, High-Reward Research program of the NIH Office of Strategic Coordination-The Common Fund offers four funding opportunities for exceptionally creative scientists at all career stages. (nih.gov)
- Am I eligible for the ReWARD program? (nih.gov)
- The ReWARD program funds (1) scientific research and (2) DEIA activities. (nih.gov)
- If you have a balance of scientific research and DEIA activities, the ReWARD program may be the best option. (nih.gov)
- Would it be wise to withdraw my proposal and apply to the NIH ReWARD program? (nih.gov)
- Or should I wait to get the decision on the proposal before moving forward to submit to the ReWARD program? (nih.gov)
- We work with you to create total reward programs, incentives, and benefits that are attractive to employees and provide an efficient return on investment. (deloitte.com)
- Rewarding Results: Aligning Incentives with High-Quality Health Care" is a national initiative of The Robert Wood Johnson Foundation (RWJF) and other funding and technical assistance partners. (nih.gov)
- The "Rewarding Results" initiative is intended to develop, evaluate, and diffuse innovations in systems of provider payments and non- financial incentives that encourage and reward high-quality care. (nih.gov)
- Reinvent your talent and reward programs around skills. (mercer.com)
- A UC Berkeley study of vulnerability reward programs, released earlier this year, found that bug bounties are cost-efficient. (darkreading.com)
- Frequent fliers generally use travel loyalty and credit card rewards programs in tandem to minimize the cost of travel. (businessinsider.com)
- Credit card rewards programs, of course, generate points on purchases. (businessinsider.com)
- Be the first to hear about new rewards, special offers and great benefits from American Express. (americanexpress.com)
- Please log in to your Membership Rewards account to redeem points or add an item to your Wish list. (americanexpress.com)
- We strongly suggest reaching out to the appropriate Scientific/Research Contact(s) listed in Part 2, Section VII of the notice of funding opportunity prior to developing an application to ensure that your plans are appropriate for ReWARD. (nih.gov)
- Past studies have found that rewarding participants during a visual perceptual task leads to performance gains. (nih.gov)
- Please note: As you are switching from this account to another Rewards account this may affect the availability of items and/or items in your shopping cart. (americanexpress.com)
- Savvy travelers can score a free vacation by using, for example, airline loyalty miles to fund a flight and credit card rewards points to pay for hotel rooms. (businessinsider.com)
- Now it's offering a new travel perk: Hilton Honors rewards points. (lifehacker.com)
- Once you've connected your Hilton Honors and Lyft accounts you'll earn Hilton Honors rewards points each time you take a ride. (lifehacker.com)
- DeLambert, who runs the blog Delta Points, is an extreme mileager -- someone who has made a full-time hobby out of gaming the travel rewards system. (businessinsider.com)
- The reward circuit links together a number of brain structures that control and regulate our ability to feel pleasure. (nih.gov)
- This requires fine-tuned regulation of brain systems that enable rapid responses to changes in the environment, such as those involved in sleep, wakefulness, stress, and reward-seeking. (nih.gov)
- The limbic system contains the brain's reward circuit or pathway. (nih.gov)
- In most endeavours in life, we get more reward for more stepping up in grade. (afr.com)
- And so the authorities are turning the mystery over to the public, appealing to historians, academics, linguists, students and hobbyists to crack the code in exchange for a €2,000 ($2,240) reward. (cnn.com)
- To this end, we shifted mood in healthy (N = 24) and depressed (N = 30) adolescents by delivering individually tailored reward prediction errors while recording magnetoencephalography (MEG) data. (nih.gov)
- For Early Stage Investigator MIRA applications it is okay to have a similar ReWARD application under review at the same time, but only one application will be funded. (nih.gov)
- For Established Investigator or New Investigator MIRA applications, it is NOT okay to have similar ReWARD application under review at the same time. (nih.gov)
- We are always improving our product offering and expanding our features and functionality to ensure you always have the very best and most rewarding mobile payment experience. (google.com)
- Can I apply for ReWARD? (nih.gov)
- We, Mysearch Global Rewards Team, comprising of vibrant youngsters excelled in ICT, having an indomitable desire and spirit in updating with the day to day Technologies emerging in the entire spectrum. (behance.net)