Polyuria
Polydipsia
Diabetes Insipidus
A disease that is characterized by frequent urination, excretion of large amounts of dilute URINE, and excessive THIRST. Etiologies of diabetes insipidus include deficiency of antidiuretic hormone (also known as ADH or VASOPRESSIN) secreted by the NEUROHYPOPHYSIS, impaired KIDNEY response to ADH, and impaired hypothalamic regulation of thirst.
Aquaporin 2
Diabetes Insipidus, Neurogenic
A genetic or acquired polyuric disorder caused by a deficiency of VASOPRESSINS secreted by the NEUROHYPOPHYSIS. Clinical signs include the excretion of large volumes of dilute URINE; HYPERNATREMIA; THIRST; and polydipsia. Etiologies include HEAD TRAUMA; surgeries and diseases involving the HYPOTHALAMUS and the PITUITARY GLAND. This disorder may also be caused by mutations of genes such as ARVP encoding vasopressin and its corresponding neurophysin (NEUROPHYSINS).
Antidiuretic Agents
Kidney Concentrating Ability
Diabetes Insipidus, Nephrogenic
A genetic or acquired polyuric disorder characterized by persistent hypotonic urine and HYPOKALEMIA. This condition is due to renal tubular insensitivity to VASOPRESSIN and failure to reduce urine volume. It may be the result of mutations of genes encoding VASOPRESSIN RECEPTORS or AQUAPORIN-2; KIDNEY DISEASES; adverse drug effects; or complications from PREGNANCY.
Deamino Arginine Vasopressin
Aquaporin 3
Aquaporin 6
Solute Carrier Family 12, Member 1
Bartter Syndrome
A group of disorders caused by defective salt reabsorption in the ascending LOOP OF HENLE. It is characterized by severe salt-wasting, HYPOKALEMIA; HYPERCALCIURIA; metabolic ALKALOSIS, and hyper-reninemic HYPERALDOSTERONISM without HYPERTENSION. There are several subtypes including ones due to mutations in the renal specific SODIUM-POTASSIUM-CHLORIDE SYMPORTERS.
Nocturia
Nocturnal Enuresis
Vasopressins
Antidiuretic hormones released by the NEUROHYPOPHYSIS of all vertebrates (structure varies with species) to regulate water balance and OSMOLARITY. In general, vasopressin is a nonapeptide consisting of a six-amino-acid ring with a cysteine 1 to cysteine 6 disulfide bridge or an octapeptide containing a CYSTINE. All mammals have arginine vasopressin except the pig with a lysine at position 8. Vasopressin, a vasoconstrictor, acts on the KIDNEY COLLECTING DUCTS to increase water reabsorption, increase blood volume and blood pressure.
Aquaporins
Arginine Vasopressin
Sodium-Potassium-Chloride Symporters
Renal Agents
Drugs used for their effects on the kidneys' regulation of body fluid composition and volume. The most commonly used are the diuretics. Also included are drugs used for their antidiuretic and uricosuric actions, for their effects on the kidneys' clearance of other drugs, and for diagnosis of renal function.
Kidney Medulla
Lithium
Lethargy
Enuresis
Involuntary discharge of URINE after expected age of completed development of urinary control. This can happen during the daytime (DIURNAL ENURESIS) while one is awake or during sleep (NOCTURNAL ENURESIS). Enuresis can be in children or in adults (as persistent primary enuresis and secondary adult-onset enuresis).
Pituitary Gland, Posterior
Neural tissue of the pituitary gland, also known as the neurohypophysis. It consists of the distal AXONS of neurons that produce VASOPRESSIN and OXYTOCIN in the SUPRAOPTIC NUCLEUS and the PARAVENTRICULAR NUCLEUS. These axons travel down through the MEDIAN EMINENCE, the hypothalamic infundibulum of the PITUITARY STALK, to the posterior lobe of the pituitary gland.
Urine
Kidney Tubules, Collecting
Osmolar Concentration
Kidney
Aquaporin 1
Neurophysins
Carrier proteins for OXYTOCIN and VASOPRESSIN. They are polypeptides of about 10-kDa, synthesized in the HYPOTHALAMUS. Neurophysin I is associated with oxytocin and neurophysin II is associated with vasopressin in their respective precursors and during transportation down the axons to the neurohypophysis (PITUITARY GLAND, POSTERIOR).
Potassium Deficiency
A condition due to decreased dietary intake of potassium, as in starvation or failure to administer in intravenous solutions, or to gastrointestinal loss in diarrhea, chronic laxative abuse, vomiting, gastric suction, or bowel diversion. Severe potassium deficiency may produce muscular weakness and lead to paralysis and respiratory failure. Muscular malfunction may result in hypoventilation, paralytic ileus, hypotension, muscle twitches, tetany, and rhabomyolysis. Nephropathy from potassium deficit impairs the concentrating mechanism, producing POLYURIA and decreased maximal urinary concentrating ability with secondary POLYDIPSIA. (Merck Manual, 16th ed)
Diuresis
Specific Gravity
Mice transgenic for an expanded CAG repeat in the Huntington's disease gene develop diabetes. (1/160)
The autosomal dominant neurological syndrome of Huntington's disease has been modeled in transgenic mice by the expression of a portion of the human huntingtin gene together with 140 CAG repeats (the R6/2 strain). The mice develop progressive chorea with onset at approximately 9 weeks of age and with death at approximately 13 weeks. Associated symptoms include weight loss and polyuria in the absence of eating or drinking deficits. We have found that these mice have insulin-responsive diabetes. Fasting glucose was 211 + 19 mg/dl in R6/2 mice compared with 93 + 5 mg/dl in C57/B6 controls (n = 12, both groups; P < 0.01). Administration of insulin intraperitoneally led to a reduction in blood glucose. At 12.5 weeks, animals were killed and pancreas weighed and analyzed for insulin and glucagon. Pancreatic mass in R6/2 mice was the same as controls, and islets appeared normal in morphology without lymphocytic infiltration. Immunohistochemical staining showed dramatic reductions in glucagon in the alpha-cells and in insulin in the beta-cells. Direct tissue assays showed glucagon and insulin content were reduced to only 10 and 15% of controls, respectively. Diabetes has been reported as being more common in Huntington's disease and other triplet repeat disorders. The R6/2 mouse should prove useful for elucidating the mechanism of diabetes in these genetic diseases. (+info)Dose related growth response to indometacin in Gitelman syndrome. (2/160)
Growth failure is a recognised feature of Gitelman syndrome, although it is not as frequent as in Bartter syndrome. Indometacin is reported to improve growth in Bartter syndrome, but not in Gitelman syndrome, where magnesium supplements are recommended. This paper presents 3 sisters with Gitelman syndrome who could not tolerate magnesium supplements, and whose hypotension and polyuria were eliminated by taking 2 mg/kg/day indometacin, but who grew poorly. However, increasing the indometacin dose to 4 mg/kg/day improved their growth significantly, without changing their symptoms or biochemistry. Gastrointestinal haemorrhage necessitated the use of misoprostol. (+info)Functional, biochemical, and molecular investigations of renal kallikrein-kinin system in diabetic rats. (3/160)
A reduction of renal kallikrein has been found in non-insulin-treated diabetic individuals, suggesting that an impaired renal kallikrein-kinin system (KKS) contributes to the development of diabetic nephropathy. We analyzed relevant components of the renal KKS in non-insulin-treated streptozotocin (STZ)-induced diabetic rats. Twelve weeks after a single injection of STZ, rats were normotensive and displayed hyperglycemia, polyuria, proteinuria, and reduced glomerular filtration rate. Blood bradykinin (BK) levels and prekallikrein activity were significantly increased compared with controls. Renal kallikrein activity was reduced by 70%, whereas urinary BK levels were increased up to threefold. Renal kininases were decreased as indicated by a 3-fold reduction in renal angiotensin-converting enzyme activity and a 1.8-fold reduction in renal expression of neutral endopeptidase 24.11. Renal cortical expression of kininogen and B2 receptors was enhanced to 1.4 and 1. 8-fold, respectively. Our data suggest that increased urinary BK levels found in severely hyperglycemic STZ-diabetic rats are related to increased filtration of components of the plasma KKS and/or renal kininogen synthesis in combination with decreased renal kinin-degrading activity. Thus, despite reduced renal kallikrein synthesis, renal KKS is activated in the advanced stage of diabetic nephropathy. (+info)Analysis of the vasopressin system and water regulation in genetically polydipsic mice. (4/160)
Polydipsic mice, STR/N, which show extreme polydipsia and polyuria, were discovered in 1958. In the STR/N, urine outputs are much higher than in control mice. The possibility of an abnormal regulation of the arginine vasopressin (AVP) system, or an abnormality in the renal susceptibility to AVP, should be considered. In this study we investigated the AVP system and water regulation in STR/N. We sequenced the AVP and the AVP V(2)-receptor genes of the STR/N by direct sequencing. No mutation was found in either of them. AVP gene expression examined by in situ hybridization and plasma sodium in 8-wk-old STR/N was significantly lower than in control mice, whereas it was significantly higher at 20 wk. Renal sensitivity to injected AVP was attenuated in 20-wk-old STR/N. The suppression of AVP synthesis due to excessive water retention in 8-wk-old STR/N suggests that polydipsia may be the primary cause in this strain. The 20-wk-old STR/N became dehydrated with the acceleration of AVP synthesis, which might have resulted from secondary desensitization to AVP. (+info)Uncompensated polyuria in a mouse model of Bartter's syndrome. (5/160)
We have used homologous recombination to disrupt the mouse gene coding for the NaK2Cl cotransporter (NKCC2) expressed in kidney epithelial cells of the thick ascending limb and macula densa. This gene is one of several that when mutated causes Bartter's syndrome in humans, a syndrome characterized by severe polyuria and electrolyte imbalance. Homozygous NKCC2-/- pups were born in expected numbers and appeared normal. However, by day 1 they showed signs of extracellular volume depletion (hematocrit 51%; wild type 37%). They subsequently failed to thrive. By day 7, they were small and markedly dehydrated and exhibited renal insufficiency, high plasma potassium, metabolic acidosis, hydronephrosis of varying severity, and high plasma renin concentrations. None survived to weaning. Treatment of -/- pups with indomethacin from day 1 prevented growth retardation and 10% treated for 3 weeks survived, although as adults they exhibited severe polyuria (10 ml/day), extreme hydronephrosis, low plasma potassium, high blood pH, hypercalciuria, and proteinuria. Wild-type mice treated with furosemide, an inhibitor of NaK2Cl cotransporters, have a phenotype similar to the indomethacin-rescued -/- adults except that hydronephrosis was mild. The polyuria, hypercalciuria, and proteinuria of the -/- adults and furosemide-treated wild-type mice were unresponsive to inhibitors of the renin angiotensin system, vasopressin, and further indomethacin. Thus absence of NKCC2 in the mouse causes polyuria that is not compensated elsewhere in the nephron. The NKCC2 mutant animals should be valuable for uncovering new pathophysiologic and therapeutic aspects of genetic disturbances in water and electrolyte recovery by the kidney. (+info)Downregulation of aquaporin-2 and -3 in aging kidney is independent of V(2) vasopressin receptor. (6/160)
The mechanisms underlying age-related polyuria were investigated in 10- and 30-mo-old female WAG/Rij rats. Urinary volume and osmolality were 3.9 +/- 0.3 ml/24 h and 2,511 +/- 54 mosmol/kgH(2)O in adult rats and 12.8 +/- 0.8 ml/24 h and 1,042 +/- 44 mosmol/kgH(2)O in senescent animals. Vasopressin V(2) receptor mRNA did not significantly differ between 10 and 30 mo, and [(3)H]vasopressin binding sites in membrane papilla were reduced by 30%. The cAMP content of the papilla was unchanged with age, whereas papillary osmolality was significantly lowered in senescent animals. The expression of aquaporin-1 (AQP1) and -4 was mostly unaltered from 10 to 30 mo. In contrast, aquaporin-2 (AQP2) and -3 (AQP3) expression was downregulated by 80 and 50%, respectively, and AQP2 was markedly redistributed into the intracellular compartment, in inner medulla of senescent animals, but not in renal cortex. These results indicate that age-related polyuria is associated with a downregulation of AQP2 and AQP3 expression in the medullary collecting duct, which is independent of vasopressin-mediated cAMP accumulation. (+info)The mechanism of acute renal failure after uranyl nitrate. (7/160)
Administration of 25 mg/kg uranyl nitrate (UN) to rats leads to a brief period of polyuria followed by progressive oliguria with death at 5 days. Factors that determine glomerular filtration rate (GFR) were examined in control Munich-Wistar rats (n equals 16) and 2 h after either 15 mg/kg (n equals 8) or 25 mg/kg (n equals 7) of UN (i.v.) utilizing direct measurements of hydrostatic and oncotic pressures and plasma flow. Total kidney GFR was reduced to 47% of control in the low dose group and to 21% in the high dose group. The simultaneous nephron filtration rate (sngfr) was 28.6 plus or minus 0.8 nl/min/g kidney wt in control, 29.1 plus or minus 1.0 in the low dose group, and 18.1 plus or minus 1.2 (P less than 0.001) in the higher dose group. This disparity in UN effect upon GFR and sngfr was due to tubular back-diffusion of solute through damaged epithelia beyond the early proximal tubule as demonstrated by microinjection of inulin and mannitol in the proximal tubule. Inulin "leak" persisted at 6 h after UN when tubular pressure had returned to normal. Comparison of sngfr measured in early vs. late proximal tubule revealed no difference after high dose UN, suggesting no significant leak of inulin from the early proximal tubule, and that the decreased sngfr was due to primary reductions in ultrafiltration. Nephron plasma flow was equal to control at both doses of UN. Also directly measured hydrostatic pressure gradient across the glomerular capillary was not changed. The effective filtration pressure achieved equilibrium in control of animals but became significantly positive at the efferent end of the capillary at both doses of UN and increased. Total glomerular permeability (LpA) was progressively reduced from control (0.089 plus or minus 0.005 nl/s/g kidney wt/mm Hg) at low dose UN (0.047 plus or minus 0.013) and high dose 0.024 plus or minus 0.003 nl/s/g kidney wt/mm Hg). Therefore UN decreases GFR by two mechanisms: (1) tubular damage leading to back-diffusion of solutes and (b) a primary reduction in sngfr due to reduced LpA. (+info)The protective effect of taurine against gentamicin-induced acute tubular necrosis in rats. (8/160)
BACKGROUND: Taurine, which is the major intracellular free beta-amino acid, is known to be an endogenous antioxidant and a membrane-stabilizing agent. In this study, we wished to know whether taurine altered the concentration of gentamicin in kidney tissue and could protect against gentamicin-induced acute proximal tubular injury. METHODS: Wistar albino rats of both sexes were assigned to three groups, which all received one of the following daily intraperitoneal injections for 8 days: (i) 0.9% sodium chloride (NaCl) alone at the same volume as gentamicin treated rats (group C; n=8); (ii) 100 mg/kg/day gentamicin alone (group G; n=8, four male, four female); or (iii) 100 mg/kg/day gentamicin plus 7.5 ml/kg/day taurine (group G+T; n=9, five male, four female). Urine was collected for 24 h for the determination of urine volume and creatinine. Intracardiac blood was collected for blood urea nitrogen (BUN) and serum creatinine determination. The kidneys were removed, weighed, and the left kidneys were subjected to biochemical analysis for the determination of thiobarbituric acid-reactive substance (TBARS) and lactate levels, and glutathione peroxidase (Gpx) and superoxide dismutase (SOD) activities. The right kidneys were divided vertically in half. The upper halves were used for histopathological examination, by light and electron microscopy. The lower halves were used to detect the gentamicin concentration within the kidney tissue, by high-performance liquid chromatography (HPLC). Changes in body weight and normalized kidney weight were recorded. RESULTS: Taurine treatment reduced gentamicin-induced increases in serum creatinine, 24 h urine volume, BUN and tissue lactate and TBARS levels (0.57+/-0.02 vs 1.06+/-0.08 mg/dl, P<0.001; 9.00+/-1.46 vs 20.9+/-2.73 ml, P<0.001; 25.3+/-1.87 vs 54.1+/-6.99 mg/dl, P<0. 001; 2.56+/-0.10 vs 3.44+/-0.08 micromol/g wet tissue, P<0.001; and 66.4+/-3.41 vs 79.5+/-5.07 nmol/g wet tissue, P>0.05, respectively). Taurine reduced the accumulation of gentamicin within the kidney tissue (233+/-29 vs 494+/-93 microg/g wet tissue, P<0.05). Taurine treatment also prevented body weight loss due to gentamicin administration (17.8+/-1.64 vs -10.0+/-7.08 g, P<0.01) and normalized reduced Gpx and SOD activities (3.46+/-0.16 vs 2.37+/-0. 15 U/g wet tissue, P<0.01; and 15577+/-377 vs 12662+/-577 U/g wet tissue, P<0.01, respectively). Light microscopic examination of the renal tissues from gentamicin-treated rats revealed severe histopathological changes, whereas specimens obtained from taurine-treated rats revealed only mild changes. This finding was supported by electron microscopic examination. CONCLUSIONS: Our observations suggest that taurine treatment attenuates the accumulation of gentamicin within kidney tissue and counteracts the deleterious effect of gentamicin on renal tubular function. They may have potentially important clinical implications. (+info)
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Polyuria
Primary polydipsia may lead to polyuria. Polyuria is usually viewed as a symptom or sign of another disorder (not a disease by ... cite book}}: ,first1= has generic name (help) "Polyuria. Medical Professional reference for Polyuria. , Patient". Patient. ... Polyuria (/ˌpɒliˈjʊəriə/) is excessive or an abnormally large production or passage of urine (greater than 2.5 L or 3 L over 24 ... Polyuria may also be due to various chemical substances, such as diuretics, caffeine, and ethanol. It may also occur after ...
Clotrimazole
Less than 1% of patients have the following side effects: burning or itching of penis of sexual partner; polyuria; vulvar ...
Chronic multifocal Langerhans cell histiocytosis
Polyuria and tuberculosis. Proceedings of the Pathological Society of Philadelphia, 1893, 16: 282-284. Archives of Pediatrics, ...
List of abnormal behaviours in animals
Excessive/submissive urination (polyuria); urinating more frequently than expected or under conditions where it would not be ...
Glossary of diabetes
Polyuria Having to urinate often; a common sign of diabetes. Postprandial blood glucose Blood taken 1-2 hours after eating to ...
Juvenile nephronophthisis
Polyuria is resistant to vasopressin. When other organ systems are affected, symptoms can include situs inversus, heart ... They include polyuria, polydipsia, weakness, and fatigue.Anemia, growth retardation, no hypertension. Proteinuria and hematuria ... Inability to conserve sodium because of defect of tubules leading to polyuria and polydipsia. Anemia is attributed to a ...
Vasopressin
It is recommended that it be cautiously used in instances of perioperative polyuria, sensitivity to the drug, asthma, seizures ... ISBN 978-1-4557-5942-2. Donaldson D (1994). "Polyuria and Disorders of Thirst". In Williams DL, Marks V (eds.). Scientific ... polyuria (excess urine production), and polydipsia (thirst). Syndrome of Inappropriate Antidiuretic Hormone secretion (SIADH) ...
Muhammad Hasanuzzaman
In addition, polyuria and heart disease occurred. On 14 April 1968, he suffered a heart attack while sitting with his son AKM ...
Endocrine gland
Hyposecretion of insulin results in diabetes mellitus; cardinal signs are polyuria, polydipsia, and polyphagia. Clarke, I. J. ( ...
Nocturia
Based on information recorded in the diary, a physician can classify the patient as having global polyuria, nocturnal polyuria ... With the 24-hour urine production within normal limits, nocturnal polyuria can be translated to having a nocturnal polyuria ... Global polyuria is the continuous overproduction of urine that is not only limited to sleep hours. Global polyuria occurs in ... Polyuria is usually viewed as a symptom or sign of another disorder (not a disease by itself), but it can be classed as a ...
Urinary system
Polyuria is a condition of excessive urine production (> 2.5 L/day). Oliguria when < 400 mL (millilitres) are produced, and ...
Laurence-Moon syndrome
Polyuria and/or polydipsia (nephrogenic diabetes insipidus) 5. Ataxia, poor coordination, imbalance 6. Mild spasticity ( ...
Central diabetes insipidus
This condition has only polyuria in common with diabetes. Although not mutually exclusive, with most typical cases, the name ... Specifically, other more common causes of polyuria and polydipsia are ruled out. Common rule outs include: diabetes mellitus, ... A better name might be "hypothalamic-neurohypophyseal ADH deficiency". Increased thirst, polyuria and dehydration with ...
Nocturnal enuresis
It is classically associated with polyuria, polydipsia, and polyphagia; weight loss, lethargy, and diaper candidiasis may also ...
Lithium orotate
Smith DF (April 1976). "Lithium orotate, carbonate and chloride: pharmacokinetics, polyuria in rats". British Journal of ...
Urine
Polyuria is a condition of excessive production of urine (> 2.5 L/day), oliguria when < 400 mL are produced, and anuria being ...
Hypoadrenocorticism in dogs
Polyuria and polydipsia, diarrhea, and shivering are occasionally reported. Hypoglycemia can also be present, and initially may ...
Frequent urination
Polyuria Nocturnal enuresis "Frequent or urgent urination: MedlinePlus Medical Encyclopedia". medlineplus.gov. 5 December 2017 ... It is often, though not necessarily, associated with urinary incontinence and polyuria (large total volume of urine). However, ...
Urinary incontinence
merck.com Polyuria: A Merck Manual of Patient Symptoms podcast. Last full review/revision September 2009 by Seyed-Ali Sadjadi, ... Age is a risk factor that increases both the severity and prevalence of UI Polyuria (excessive urine production) of which, in ... Polyuria generally causes urinary urgency and frequency, but does not necessarily lead to incontinence. Neurogenic disorders ... Non-urologic causes may include infection, medication or drugs, psychological factors, polyuria, hydrocephalus, stool impaction ...
Medical history
... polyuria, increased appetite (polyphagia) and irritability). Musculoskeletal system (any bone or joint pain accompanied by ...
Diabetes insipidus
With increased thirst, the person now experiences a polydipsia and polyuria cycle. Hereditary forms of diabetes insipidus ... and traditionally have administered thiazide diuretics for lithium-induced polyuria and nephrogenic diabetes insipidus. However ...
Bassia scoparia
Livestock ingesting large amounts can experience weight loss, hyperbilirubinemia, photosensitization, and polyuria. The plant's ...
Hypercalcaemia
Observable symptoms may develop such as polydipsia, polyuria, extreme fatigue, or constipation. In certain outdoor environments ...
Nephrogenic diabetes insipidus
Polyuria will continue as long as the patient is able to drink. If the patient is unable to drink and is still unable to ... The two diseases were named (in ancient times) for the fact that one features polyuria in which the urine tastes sweet, whereas ... The major causes of acquired nephrogenic diabetes insipidus that produce clinical symptoms (e.g., polyuria) in the adult are ... diabetes mellitus causes polyuria via osmotic diuresis, due to the high blood sugar leaking into the urine, taking excess water ...
Fraley syndrome
Patients may also present with a fever, nausea, vomiting, dysuria and polyuria. If untreated, the chronic infections can ...
Antimanic drugs
This gives rise to diabetes insipidus, a disorder characterized by polyuria and polydipsia. Other adverse effects of lithium ...
Polydipsia
The combination of polydipsia and (nocturnal) polyuria is also seen in (primary) hyperaldosteronism (which often goes with ... As it is often accompanied by polyuria (excessive urination) and low sodium levels. Investigations directed at diagnosing ...
History of diabetes
Polyuria in diabetes was associated with a sweet taste in Sanskrit texts of the 5th/6th century BC, at the time of two notable ... In 1920, Jean Camus and Gustave Roussy summarized a number of years of research, reporting that they had produced polyuria in ... Greco-Roman accounts of what we now know as diabetes primarily describe excessive urination (polyuria). There is no known ... He excised the pancreas of a neighbour's hunting dog, causing polyuria and polydipsia. Brunner very clearly described these ...
Insomnia
Nocturnal polyuria, excessive nighttime urination, can also result in a poor quality of sleep. Some cases of insomnia are not ...
Primary hyperparathyroidism
"Thrones" refers to polyuria and constipation "Psychiatric overtones" refers to effects on the central nervous system. Symptoms ... polyuria and polydipsia). These can ultimately lead to kidney failure. "Bones" refers to bone-related complications. The ...
No data available that match "polyuria"
PolyphagiaThirstNocturiaUrinationPolydipsia and PolyuriaSymptomsDysuriaNocturnalOliguriaPolydypsiaUrine outputClinicalMetabolicBlood SugarAmountsGlucoseRenalTreatmentExperiencePatientCaseUrinationThirstRenalPolydipsia and polyuriaAbnormally large volumesHematuriaCause polyuriaCauses PolyuriaFatigueDiarrhoeaMuscle weaknessCentral diabetesDiabetes mellitusWeaknessChronicMildAcidosisNatriuresisInabilityWeightClinicalMicturitionCoatingsOnsetHighTypeAdverseDiagnoseSigns of diabetesIntake
Polyphagia3
- One of the three cardinal signs of diabetes is polyuria, the other two being polyphagia (increased hunger) and polydipsia (increased thirst). (mypetneedsthat.com)
- The high plasma glucose produces the classical symptoms of polyuria, polydipsia and polyphagia (Rother, 2007). (researchwap.net)
- The classic symptoms of diabetes are polyuria (frequent urination), polydipsia (increased thirst), polyphagia (increased hunger), and weight loss . (thediabeticpharmacist.com)
Thirst2
- Polydipsia is a condition characterized by an increase in thirst, due to dehydration caused due to excess loss of water from the body due to Polyuria.The thirst increases exceptionally in this condition. (avedaayur.com)
- Polyuria, excessive hunger, extreme thirst and unintentional weight lost are typical symptoms 2,3 . (bvsalud.org)
Nocturia3
- In this case, it is called nocturnal polyuria (or nocturia). (jpeeiclinic.com)
- More specifically, NOPIA researches nocturia, nocturnal incontinence and bedwetting, nocturnal retention and nocturnal polyuria. (ugent.be)
- This condition leads to increased urinary frequency and volume (eg, polyuria), which is particularly troublesome at night (eg, nocturia) and often leads to enuresis in a previously continent child. (medscape.com)
Urination4
- The causes of polyuria or frequent urination are many such as diabetes mellitus, pregnancy, urinary tract infections , prostate conditions, side effects of certain medicines and antibiotics, etc. (jiva.com)
- Excessive urination volume (or polyuria) occurs when you urinate more than normal. (jpeeiclinic.com)
- As such, we shall try to categorize the causes of excessive urination in cats into pollakiuria and polyuria. (mypetneedsthat.com)
- At that time, physicians were distinguishing different types of diabetes (diseases characterized by polyuria, excessive urination) and they knew of a rare disease called acromegaly, characterized by enlarged body parts. (visionlearning.com)
Polydipsia and Polyuria1
- Nephrogenic diabetes insipidus resulting in polydipsia and polyuria is seen in about 20% of patients. (clevelandclinicmeded.com)
Symptoms4
- Very few people know that fenugreek seeds can help control polyuria, one of the main symptoms of Type 1 diabetes and Type 2 diabetes. (jiva.com)
- However, one of "the most common" diabetes symptoms experienced by "many people" with the blood sugar condition is polyuria, the charity explains. (fitnessblogger.info)
- Similarly to other diabetes symptoms, polyuria is triggered by "abnormally high" blood sugar levels. (fitnessblogger.info)
- While polyuria is one of the most "common" symptoms of diabetes, it isn't the only red flag. (fitnessblogger.info)
Dysuria2
- However, if 24-hour water intake is normal, pathologic PU and PD are unlikely and another inciting factor (e.g., hot weather) should be sought or misinterpretation of polyuria (e.g., dysuria instead of polyuria) should be considered. (vin.com)
- It is used to treat the insrfficiency syndrome of kidney yang marked by lumbago, pedal flaccidity, cold sensation in the lower part of the body, cpasm in the lower abdomen, dysuria or polyuria, weak and small pulse in the chi part, pale and enlarged tongue with thin and white fur. (tcmherbsusa.com)
Nocturnal1
- There is some overlap with the research on nocturnal polyuria. (ugent.be)
Oliguria1
- Mild oliguria (380 mL/24 h) and polyuria rodents. (cdc.gov)
Polydypsia1
- polyuria (PU) -an overproduction of urine-goes hand-in-paw with polydypsia (PD) , excessive water intake. (wagglemail.com)
Urine output1
- Given our example above, if you expect your kitty to be voiding about 120 mL in a 24-hour period, then polyuria will mean a urine output that is at least 10 percent greater than the expected norm. (mypetneedsthat.com)
Clinical1
- Dysfunction in any of these areas results in the clinical signs of polyuria (PU) and polydipsia (PD). (vin.com)
Metabolic1
- This can occur due to many factors including excessive cold or physical activity, disease states that heighten the metabolic rate, insufficient calories or poor diet, inability to ingest the diet, inability to digest and/or absorb the nutrients in the diet, and loss of nutrients from vomiting, diarrhea, or polyuria. (companionpetvet.com)
Blood Sugar2
- In uncontrolled diabetes, polyuria occurs because of high blood sugar levels. (akronchildrens.org)
- As polyuria is driven by an increase in blood glucose, this sign can also crop up once you're diagnosed with the blood sugar condition. (fitnessblogger.info)
Amounts2
- A person with polyuria makes large amounts of urine. (akronchildrens.org)
- Polyuria, or peeing more frequently than usual, details passing excessive amounts of urine each time you pee. (fitnessblogger.info)
Glucose1
- Your doctor will measure your blood glucose if they suspect that diabetes is causing your polyuria. (jpeeiclinic.com)
Renal2
- As per the words of Bhasin & Velez (2016), polyuria is the excessive flow of urine or the enhancement of the renal output of an individual which can occur for multiple reasons. (myassignmentservices.com)
- Renal tubular dysfunction ranges from isolated renal tubular acidosis to full-blown Fanconi syndrome (polyuria, aminoaciduria, glycosuria, phosphaturia, and bicarbonate loss). (altmeyers.org)
Treatment1
- Lithium-Induced Polyuria: May develop during initiation of treatment. (nih.gov)
Experience2
- You will definitely experience great relief from polyuria. (jiva.com)
- You may experience polyuria as a side effect of these medications. (jpeeiclinic.com)
Patient1
- It has been identified that the patient has the issue of polyuria. (myassignmentservices.com)
Case1
- It might be a case of polyuria. (jiva.com)
Urination5
- Polyuria (PU) and polydipsia (PD) refer to excessive urination and excessive drinking, respectively. (vetsecure.com)
- Polyuria (PU) and polydipsia (PD) are the medical terms used to describe excessive urination and excessive drinking, respectively. (vetsecure.com)
- Symptoms include excessive drinking (polydipsia), excessive urination (polyuria), and glucose in the urine (glucosuria. (caninegeneticdiseases.net)
- Diabetes generally appears as polyuria (frequent urination) and polydipsia (increased thirst). (diabetesandrelatedhealthissues.com)
- Diabetes insipidus is simply excessive urination polyuria and complications thereof, caused by an antidiuretice hormone called a vasopressin. (stabilpartner.hu)
Thirst4
- Early symptoms of diabetes might include polyuria, thirst or fatigue. (bpac.org.nz)
- Clinical fetaures are polydipsia, polyphagia and polyuria which result from hyperglycemia-induced osmotic diuresis and secondary thirst. (abcam.com)
- Thirst, polydipsia, and polyuria appeared 2 days after the second vaccination. (cdc.gov)
- Polyuria, excessive hunger, extreme thirst and unintentional weight lost are typical symptoms 2,3 . (bvsalud.org)
Renal4
- Disturbances in these regulatory mechanisms can lead to polyuria-polydipsia syndrome, which comprises of three different conditions: central diabetes insipidus (DI) due to insufficient secretion of AVP, nephrogenic DI caused by renal insensitivity to AVP action and primary polydipsia due to excessive fluid intake and consequent physiological suppression of AVP. (nih.gov)
- Both patients had polydipsia, polyuria, and markedly reduced renal function. (fluoridealert.org)
- Polyuria, acute renal failure, nephrocalcinosis. (mims.com)
- Disrupted function or regulation of AQP2 or the AVPR2 results in nephrogenic diabetes insipidus NDIa common clinical condition of renal origin characterized by polydipsia and polyuria. (stabilpartner.hu)
Polydipsia and polyuria2
- You may hear your vet use the words polydipsia and polyuria (PUPD for short). (pdsa.org.uk)
- The only symptoms in primary central diabetes insipidus are polydipsia and polyuria. (stabilpartner.hu)
Abnormally large volumes1
- Polyuria (producing abnormally large volumes of urine)- Usually the kidneys reabsorb all the sugar in the body. (dlife.com)
Hematuria1
- Later signs are associated with kidney and liver damage and include dehydration, icterus, hematuria and polyuria. (dvm360.com)
Cause polyuria2
- Fortunately, most conditions that cause polyuria and polydipsia are manageable or curable. (vetsecure.com)
- Diabetes mellitus ("sugar diabetes ") is unrelated to diabetes insipidus except that they can both cause polyuria. (hopkinsguides.com)
Causes Polyuria1
- What Causes Polyuria? (webmd.com)
Fatigue1
- A few days before the fourth cycle, the patient complained of intense headaches, profound fatigue, nocturia, polyuria (up to 10 L urine/daily), and polydipsia. (frontiersin.org)
Diarrhoea1
- Has the patient had recent diarrhoea, vomiting or extreme polyuria? (endobible.com)
Muscle weakness1
- Small, frequent bolus feedings often are grouped by protein allergens ige mediated allergic reactions infusion reactions, anaphylaxis in up to weeks, although may also be monitored closely in these infants will respond to the spinal nerve roots, dorsal root ganglion bilateral involvement o proximal muscle weakness, polyuria, electrocardiogram ecg is indicated i it can occur when these agents occur in the late s with microscopy, urine sodium, urea, and tolvaptan. (easternpropane.com)
Central diabetes1
- The major symptoms of central diabetes insipidus DI are polyuria, nocturia, and polydipsia due to the diabetes insipidus test defect. (stabilpartner.hu)
Diabetes mellitus1
- Introduction: Polyuria is a sign for many disease processes, including diabetes mellitus and diabetes insipidus. (scirp.org)
Weakness1
- While hyperglycemic emergencies are likely extremely rare in patients receiving COVID-19 vaccinations, it is important for clinicians to be aware of these complications and suspect severe hyperglycemia in those who develop postvaccination polyuria, polydipsia, blurry vision, weakness, and even confusion, among other symptoms. (pinedaleonline.com)
Chronic1
- I have also hypothyroidism (Thyroid Erfa) and cervical muscular dystonia, chronic sinusitis, periodical atrial fibrillation, arthrosis and possibly reactive arthritis, sometimes polyuria, and an unknown infiltration in my lung. (medhelp.org)
Mild1
- Mild oliguria (380 mL/24 h) and polyuria rodents. (cdc.gov)
Acidosis2
Natriuresis3
- Mechanism of polyuria and natriuresis in atrioventricular nodal tachycardia. (bmj.com)
- In a cohort of ADPKD on tolvaptan, we aimed to identify the determinants of polyuria, to evaluate the impact of dietary counselling on the natriuresis and to compare the decline of estimated glomerular filtration rate (eGFR) before and after tolvaptan. (asn-online.org)
- Natriuresis before tolvaptan is the major determinant of the polyuria on tolvaptan, but natriuresis can be significantly reduced by dietetic counselling. (asn-online.org)
Inability1
- This can occur due to many factors including excessive cold or physical activity, disease states that heighten the metabolic rate, insufficient calories or poor diet, inability to ingest the diet, inability to digest and/or absorb the nutrients in the diet, and loss of nutrients from vomiting, diarrhea, or polyuria. (allcreaturesanimalhospital-hs.com)
Weight2
- Case Presentation: We report a case of a 48-year-old woman who presented with polyuria, polydipsia, nocturia, and weight loss after a return from a visit to Russia, during which she received a five-day course of antibiotic Demeclocycline, a tetracycline derivative for dental treatment. (scirp.org)
- Classic symptoms (polydipsia, polyuria and weight loss) were present in around half of the participants but were not more prevalent in those with diabetes. (who.int)
Clinical1
- What Are the Clinical Signs of Polyuria and Polydipsia? (vetsecure.com)
Micturition1
- Polyuria, incontinence, and difficulty in micturition may occur. (inchem.org)
Coatings2
- Diamines are majorly consumed in epoxy coatings, polyuria, and adhesives & sealants applications. (aggregateresearch.com)
- LINE-X ULTRA is an aliphatic polyuria which is sprayed thinner than elastomer coatings can provide superior strength and reliability. (thomasnet.com)
Onset2
- Generally, lithium nephrotoxicity will occur within a month of onset of use of the drug, manifested predominantly by polyuria and polydipsia. (medscape.com)
- Patients on ICIs and their caregivers should be informed about that risk and be empowered to alert the referring specialists early, at the onset of panhypopituitarism symptoms, including polyuria/polydipsia. (frontiersin.org)
High1
- Salient features middle aged obese polyuria and high mortality. (aaan.org)
Type1
- Ces vingt dernières années, on assiste à une augmentation spectaculaire du nombre de cas de diabète de type 1 avec une mortalité plus élevée en Afrique Noire en lien avec les difficultés d'accès aux soins, à la rupture de suivi engendrant un nombre élevé de perdus de vue. (bvsalud.org)
Adverse1
- With the exception of 1 voriconazole-treated parrot that developed polyuria, adverse effects were not evident. (avma.org)
Diagnose1
- Your doctor looks at many things to diagnose polyuria. (webmd.com)
Signs of diabetes1
- Polyuria is often one of the first signs of diabetes . (webmd.com)
Intake2
- Polyuria is a significant side-effect that can be attenuated by reducing daily sodium intake. (asn-online.org)
- For each baseline increment of 10 mmol of daily sodium intake the odds ratio of polyuria was 3.4 (95%CI: 2.7-4.5). (asn-online.org)