Polytetrafluoroethylene
Blood Vessel Prosthesis
Polyethylene Terephthalates
Blood Vessel Prosthesis Implantation
Arteriovenous Shunt, Surgical
Popliteal Artery
Anastomosis, Surgical
Prostheses and Implants
Coated Materials, Biocompatible
Biocompatible Materials
Suture Techniques
Polyurethanes
Jugular Veins
Tibial Arteries
Surgical Mesh
Seroma
Reoperation
Hyperplasia
Vascular Grafting
Fibrin Tissue Adhesive
Femoral Vein
Dura Mater
Iliac Artery
Arterial Occlusive Diseases
Axillary Artery
Catheters, Indwelling
Materials Testing
Silicone Elastomers
Postoperative Complications
Tunica Intima
Renal Dialysis
Hernia, Ventral
Polypropylenes
Aorta, Abdominal
Life Tables
Ischemia
Abdominal Wall
Membranes, Artificial
Treatment Outcome
Alloys
Stents
Chordae Tendineae
Inguinal Canal
Limb Salvage
Dogs
Follow-Up Studies
Superior Vena Cava Syndrome
Aneurysm
Implants, Experimental
Papio
Axillary Vein
Constriction, Pathologic
Endarterectomy, Carotid
Heart-Lung Machine
Prosthesis Failure
Ultrasonography, Doppler, Duplex
Scleral Diseases
Diaphragmatic Eventration
Transplants
Vena Cava, Inferior
Prospective Studies
Microscopy, Electron, Scanning
Tissue Adhesives
Lower Extremity
Angioplasty
Tensile Strength
Silicones
Reconstructive Surgical Procedures
Vascular Neoplasms
Thrombectomy
Surface Properties
Angioplasty, Balloon
Carotid Stenosis
Pericardium
Aortic Aneurysm, Abdominal
Bacteriophage inactivation at the air-water-solid interface in dynamic batch systems. (1/882)
Bacteriophages have been widely used as surrogates for human enteric viruses in many studies on virus transport and fate. In this investigation, the fates of three bacteriophages, MS2, R17, and phiX174, were studied in a series of dynamic batch experiments. Both MS2 and R17 readily underwent inactivation in batch experiments where solutions of each phage were percolated through tubes packed with varying ratios of glass and Teflon beads. MS2 and R17 inactivation was the result of exposure to destructive forces at the dynamic air-water-solid interface. phiX174, however, did not undergo inactivation in similar studies, suggesting that this phage does not accumulate at air-water interfaces or is not affected by interfacial forces in the same manner. Other batch experiments showed that MS2 and R17 were increasingly inactivated during mixing in polypropylene tubes as the ionic strength of the solution was raised (phiX174 was not affected). By the addition of Tween 80 to suspensions of MS2 and R17, phage inactivation was prevented. Our data suggest that viral inactivation in simple dynamic batch experiments is dependent upon (i) the presence of a dynamic air-water-solid interface (where the solid is a hydrophobic surface), (ii) the ionic strength of the solution, (iii) the concentration of surface active compounds in the solution, and (iv) the type of virus used. (+info)Association of plasma fibrinogen concentration with vascular access failure in hemodialysis patients. (2/882)
BACKGROUND: Elevated plasma fibrinogen is an important risk factor for coronary artery disease in the general population and patients with chronic renal failure. High plasma fibrinogen may trigger thrombus formation in arteriovenous fistulas. We performed a prospective, cohort study to evaluate the association of plasma fibrinogen concentration with vascular access failure in patients undergoing long-term haemodialysis. METHODS: Between September 1989 and October 1995, 144 patients underwent a vascular access operation. In March 1997, 102 patients (56 M, 46 F) who had been followed up for more than 18 months (median; 37 months, range; 18-102 months) were included in the study. The median age of the patients was 52 years (range; 19-78 years). In 35 patients, renal disease was secondary to diabetes mellitus. The type of vascular access was a polytetrafluoroethylene (PTFE) graft in 17 patients. Seventy-seven patients received recombinant human erythropoietin (r-HuEPO) therapy during the follow-up period. Plasma fibrinogen, albumin, total cholesterol, hematocrit, platelets and creatinine were measured at the time of operation. Vascular access failure was defined as the occurrence of complications requiring transluminal angioplasty, thrombolytic therapy or surgical repair. RESULTS: Thirty-eight patients had at least one vascular access failure and the incidence was 0.3 (range; 0-2.4) episodes per patient-year. The survival rate of vascular access was 78% (native fistula; 80%, PTFE graft; 71%) after 12 months and 70% (native fistula; 73%, PTFE graft; 51%) after 24 months. Older age, a PTFE graft, r-HuEPO therapy, higher hematocrit, lower albumin and higher fibrinogen levels were significantly associated with vascular access failure, whereas gender, diabetes mellitus, total cholesterol and platelet count were not. Plasma fibrinogen was inversely correlated with albumin (r=-0.38, P=0.001). The cumulative vascular access survival was significantly lower in patients with high plasma fibrinogen levels (> or = 460 mg/dl) compared with patients with low levels (< 460 mg/dl) (P=0.007). Independent risk factors for vascular access failure analysed by Cox's proportional hazards model were older age (RR; 1.36 by 10-year increment), higher fibrinogen level (RR; 1.20 by 100 mg/dl increment), PTFE graft (RR; 2.28) and r-HuEPO therapy (RR; 3.79). CONCLUSION: High plasma fibrinogen level is an independent risk factor for vascular access failure in haemodialysis patients. (+info)Isolated femoropopliteal bypass graft for limb salvage after failed tibial reconstruction: a viable alternative to amputation. (3/882)
PURPOSE: Femoropopliteal bypass grafting procedures performed to isolated popliteal arteries after failure of a previous tibial reconstruction were studied. The results were compared with those of a study of primary isolated femoropopliteal bypass grafts (IFPBs). METHODS: IFPBs were only constructed if the uninvolved or patent popliteal segment measured at least 7 cm in length and had at least one major collateral supplying the calf. When IFPB was performed for ischemic lesions, these lesions were usually limited to the digits or small portions of the foot. Forty-seven polytetrafluoroethylene grafts and three autogenous reversed saphenous vein grafts were used. RESULTS: Ankle brachial pressure index (ABI) increased after bypass grafting by a mean of 0.46. Three-year primary life table patency and limb-salvage rates for primary IFPBs were 73% and 86%, respectively. All eight IFPBs performed after failed tibial bypass grafts remained patent for 2 to 44 months, with patients having viable, healed feet. CONCLUSION: In the presence of a suitable popliteal artery and limited tissue necrosis, IFPB can have acceptable patency and limb-salvage rates, even when a polytetrafluoroethylene graft is used. Secondary IFPB can be used to achieve limb salvage after failed tibial bypass grafting. (+info)Right atrial bypass grafting for central venous obstruction associated with dialysis access: another treatment option. (4/882)
PURPOSE: Central venous obstruction is a common problem in patients with chronic renal failure who undergo maintenance hemodialysis. We studied the use of right atrial bypass grafting in nine cases of central venous obstruction associated with upper extremity venous hypertension. To better understand the options for managing this condition, we discuss the roles of surgery and percutaneous transluminal angioplasty with stent placement. METHODS: All patients had previously undergone placement of bilateral temporary subclavian vein dialysis catheters. Severe arm swelling, graft thrombosis, or graft malfunction developed because of central venous stenosis or obstruction in the absence of alternative access sites. A large-diameter (10 to 16 mm) externally reinforced polytetrafluoroethylene (GoreTex) graft was used to bypass the obstructed vein and was anastomosed to the right atrial appendage. This technique was used to bypass six lesions in the subclavian vein, two lesions at the innominate vein/superior vena caval junction, and one lesion in the distal axillary vein. RESULTS: All patients except one had significant resolution of symptoms without operative mortality. Bypass grafts remained patent, allowing the arteriovenous grafts to provide functional access for 1.5 to 52 months (mean, 15.4 months) after surgery. CONCLUSION: Because no mortality directly resulted from the procedure and the morbidity rate was acceptable, this bypass grafting technique was adequate in maintaining the dialysis access needed by these patients. Because of the magnitude of the procedure, we recommend it only for the occasional patient in whom all other access sites are exhausted and in whom percutaneous dilation and/or stenting has failed. (+info)Infrarenal endoluminal bifurcated stent graft infected with Listeria monocytogenes. (5/882)
Prosthetic graft infection as a result of Listeria monocytogenes is an extremely rare event that recently occurred in a 77-year-old man who underwent endoluminal stent grafting for infrarenal abdominal aortic aneurysm. The infected aortic endoluminal prosthesis was removed by means of en bloc resection of the aneurysm and contained endograft with in situ aortoiliac reconstruction. At the 10-month follow-up examination, the patient was well and had no signs of infection. (+info)Prospective randomised trial of distal arteriovenous fistula as an adjunct to femoro-infrapopliteal PTFE bypass. (6/882)
OBJECTIVES: To compare graft patency and limb salvage rate following femoro-infrapopliteal bypass using ePTFE grafts with and without the addition of adjuvant arterio-venous fistula. DESIGN: A prospectively randomised controlled trial. MATERIALS: Patients referred to two teaching hospital vascular surgery units in the U.K. for the treatment of critical limb ischaemia. METHODS: Eighty-seven patients (M:F; 2.3:1) undergoing 89 femoro-intrapopliteal bypass operations with ePTFE grafts for critical limb ischaemia were randomly allocated to have AVF included in the operative procedure (n = 48) or to a control group without AVF (n = 41). An interposition vein-cuff was incorporated at the distal anastomosis in all patients. RESULTS: The cumulative rates of primary patency and limb salvage at 1-year after operation for patients with AVF were 55.2% and 54.1% compared to 53.4% and 43.2%, respectively, for the control group. The differences between the AVF and control groups did not reach statistical significance, in terms of either graft patency or limb salvage, at any stage after operation (Log-Rank test). CONCLUSIONS: AVF confers no additional significant clinical advantage over interposition vein cuff in patients having femoro-infrapopliteal bypass with ePTFE grants for critical limb ischaemia. (+info)Bypass graft of an occluded inferior vena cava: report of a case with patency at five years. (7/882)
Venous reconstructive surgery for chronic occlusive disease has evolved slower than its arterial counterpart. Factors intrinsic to the venous system that have been implicated in discouraging experimental and clinical results include enhanced graft thrombogenicity, low velocity of blood flow, and wall collapsibility. 1,2 We present a case of a 24-year-old man with symptomatic occlusion of the inferior vena cava, treated with a prosthetic bypass graft to the supra diaphragmatic cava. The graft was patent 5 years later, and the patient remained asymptomatic. (+info)Posterior approach to the deep femoral artery. (8/882)
Unusual surgical approaches to the deep femoral artery are valuable when the standard anterior approach is difficult because of scarring or infection. A posterior approach to the deep femoral artery in patients, in whom all other approaches were unsuitable, is described. (+info)Graft occlusion can occur due to a variety of factors, including:
1. Blood clots forming within the graft
2. Inflammation or infection within the graft
3. Narrowing or stenosis of the graft
4. Disruption of the graft material
5. Poor blood flow through the graft
The signs and symptoms of vascular graft occlusion can vary depending on the location and severity of the blockage. They may include:
1. Pain or tenderness in the affected limb
2. Swelling or redness in the affected limb
3. Weakness or numbness in the affected limb
4. Difficulty walking or moving the affected limb
5. Coolness or discoloration of the skin in the affected limb
If you experience any of these symptoms, it is important to seek medical attention as soon as possible. A healthcare professional can diagnose vascular graft occlusion using imaging tests such as ultrasound, angiography, or MRI. Treatment options for vascular graft occlusion may include:
1. Medications to dissolve blood clots or reduce inflammation
2. Surgical intervention to repair or replace the graft
3. Balloon angioplasty or stenting to open up the blocked graft
4. Hyperbaric oxygen therapy to improve blood flow and promote healing.
Preventive measures to reduce the risk of vascular graft occlusion include:
1. Proper wound care and infection prevention after surgery
2. Regular follow-up appointments with your healthcare provider
3. Avoiding smoking and other cardiovascular risk factors
4. Taking medications as directed by your healthcare provider to prevent blood clots and inflammation.
It is important to note that vascular graft occlusion can be a serious complication after surgery, but with prompt medical attention and appropriate treatment, the outcome can be improved.
Examples of how 'Tissue Adhesions' is used in the medical field:
1. In gastrointestinal surgery, tissue adhesions can form between the intestines and other organs, leading to bowel obstruction, inflammation, or other complications.
2. In cardiovascular surgery, tissue adhesions can form between the heart and surrounding tissues, causing impaired heart function and increasing the risk of postoperative complications.
3. In gynecological surgery, tissue adhesions can form between the uterus and other pelvic organs, leading to pain, bleeding, and infertility.
4. In oncologic surgery, tissue adhesions can form between cancerous tissues and surrounding normal tissues, making it difficult to remove the tumor completely.
5. In chronic diseases such as endometriosis, tissue adhesions can form between the uterus and other pelvic structures, leading to pain and infertility.
6. Tissue adhesions can also form within the skin, causing keloids or other types of scarring.
Treatment options for tissue adhesions depend on the location, size, and severity of the adhesions, as well as the underlying cause. Some common treatment options include:
1. Surgical removal of adhesions: This involves surgically removing the fibrous bands or scar tissue that are causing the adhesions.
2. Steroid injections: Injecting steroids into the affected area can help reduce inflammation and shrink the adhesions.
3. Physical therapy: Gentle stretching and exercise can help improve range of motion and reduce stiffness in the affected area.
4. Radiofrequency ablation: This is a minimally invasive procedure that uses heat to break down and remove the fibrous bands causing the adhesions.
5. Laser therapy: Laser therapy can be used to break down and remove the fibrous bands causing the adhesions, or to reduce inflammation and promote healing.
6. Natural remedies: Some natural remedies such as turmeric, ginger, and omega-3 fatty acids have anti-inflammatory properties and may help reduce inflammation and improve symptoms.
Preventing tissue adhesions is not always possible, but there are some measures that can be taken to reduce the risk of their formation. These include:
1. Proper wound care: Keeping wounds clean and dry, and using sterile dressings can help prevent infection and reduce the risk of adhesion formation.
2. Minimizing trauma: Avoiding unnecessary trauma to the affected area can help reduce the risk of adhesion formation.
3. Gentle exercise: Gentle exercise and stretching after surgery or injury can help improve range of motion and reduce stiffness in the affected area.
4. Early mobilization: Early mobilization after surgery or injury can help reduce the risk of adhesion formation.
5. Avoiding smoking: Smoking can impede wound healing and increase the risk of adhesion formation, so avoiding smoking is recommended.
6. Using anti-adhesive agents: Applying anti-adhesive agents such as silicone or hydrogel to the affected area after surgery or injury can help reduce the risk of adhesion formation.
It's important to note that the most effective method for preventing or treating tissue adhesions will depend on the specific cause and location of the adhesions, as well as the individual patient's needs and medical history. A healthcare professional should be consulted for proper evaluation and treatment.
The definition of 'seroma' has been used in medical literature to describe a type of postoperative complication that is common after surgery, particularly after thoracic and abdominal procedures. Seromas are often diagnosed based on physical examination findings such as swelling, pain, and limited range of motion. Imaging studies such as ultrasound or CT scans may be used to confirm the presence of a seroma and to rule out other possible causes of swelling. Treatment options for seromas include draining the fluid with a needle or surgically removing the sac. In some cases, antibiotics may be prescribed if there is evidence of infection.
Seroma is a term that is used frequently in medical literature and should be familiar to healthcare professionals who work in surgical settings, particularly those who specialize in thoracic and abdominal procedures. Understanding the definition and clinical features of seromas can help healthcare providers provide appropriate diagnosis and management for patients who develop this complication after surgery.
There are different types of hyperplasia, depending on the location and cause of the condition. Some examples include:
1. Benign hyperplasia: This type of hyperplasia is non-cancerous and does not spread to other parts of the body. It can occur in various tissues and organs, such as the uterus (fibroids), breast tissue (fibrocystic changes), or prostate gland (benign prostatic hyperplasia).
2. Malignant hyperplasia: This type of hyperplasia is cancerous and can invade nearby tissues and organs, leading to serious health problems. Examples include skin cancer, breast cancer, and colon cancer.
3. Hyperplastic polyps: These are abnormal growths that occur in the gastrointestinal tract and can be precancerous.
4. Adenomatous hyperplasia: This type of hyperplasia is characterized by an increase in the number of glandular cells in a specific organ, such as the colon or breast. It can be a precursor to cancer.
The symptoms of hyperplasia depend on the location and severity of the condition. In general, they may include:
* Enlargement or swelling of the affected tissue or organ
* Pain or discomfort in the affected area
* Abnormal bleeding or discharge
* Changes in bowel or bladder habits
* Unexplained weight loss or gain
Hyperplasia is diagnosed through a combination of physical examination, imaging tests such as ultrasound or MRI, and biopsy. Treatment options depend on the underlying cause and severity of the condition, and may include medication, surgery, or other interventions.
Types of Arterial Occlusive Diseases:
1. Atherosclerosis: Atherosclerosis is a condition where plaque builds up inside the arteries, leading to narrowing or blockages that can restrict blood flow to certain areas of the body.
2. Peripheral Artery Disease (PAD): PAD is a condition where the blood vessels in the legs and arms become narrowed or blocked, leading to pain or cramping in the affected limbs.
3. Coronary Artery Disease (CAD): CAD is a condition where the coronary arteries, which supply blood to the heart, become narrowed or blocked, leading to chest pain or a heart attack.
4. Carotid Artery Disease: Carotid artery disease is a condition where the carotid arteries, which supply blood to the brain, become narrowed or blocked, leading to stroke or mini-stroke.
5. Renal Artery Stenosis: Renal artery stenosis is a condition where the blood vessels that supply the kidneys become narrowed or blocked, leading to high blood pressure and decreased kidney function.
Symptoms of Arterial Occlusive Diseases:
1. Pain or cramping in the affected limbs
2. Weakness or fatigue
3. Difficulty walking or standing
4. Chest pain or discomfort
5. Shortness of breath
6. Dizziness or lightheadedness
7. Stroke or mini-stroke
Treatment for Arterial Occlusive Diseases:
1. Medications: Medications such as blood thinners, cholesterol-lowering drugs, and blood pressure medications may be prescribed to treat arterial occlusive diseases.
2. Lifestyle Changes: Lifestyle changes such as quitting smoking, exercising regularly, and eating a healthy diet can help manage symptoms and slow the progression of the disease.
3. Endovascular Procedures: Endovascular procedures such as angioplasty and stenting may be performed to open up narrowed or blocked blood vessels.
4. Surgery: In some cases, surgery may be necessary to treat arterial occlusive diseases, such as bypass surgery or carotid endarterectomy.
Prevention of Arterial Occlusive Diseases:
1. Maintain a healthy diet and lifestyle
2. Quit smoking and avoid exposure to secondhand smoke
3. Exercise regularly
4. Manage high blood pressure, high cholesterol, and diabetes
5. Avoid excessive alcohol consumption
6. Get regular check-ups with your healthcare provider
Early detection and treatment of arterial occlusive diseases can help manage symptoms, slow the progression of the disease, and prevent complications such as heart attack or stroke.
1. Infection: Bacterial or viral infections can develop after surgery, potentially leading to sepsis or organ failure.
2. Adhesions: Scar tissue can form during the healing process, which can cause bowel obstruction, chronic pain, or other complications.
3. Wound complications: Incisional hernias, wound dehiscence (separation of the wound edges), and wound infections can occur.
4. Respiratory problems: Pneumonia, respiratory failure, and atelectasis (collapsed lung) can develop after surgery, particularly in older adults or those with pre-existing respiratory conditions.
5. Cardiovascular complications: Myocardial infarction (heart attack), cardiac arrhythmias, and cardiac failure can occur after surgery, especially in high-risk patients.
6. Renal (kidney) problems: Acute kidney injury or chronic kidney disease can develop postoperatively, particularly in patients with pre-existing renal impairment.
7. Neurological complications: Stroke, seizures, and neuropraxia (nerve damage) can occur after surgery, especially in patients with pre-existing neurological conditions.
8. Pulmonary embolism: Blood clots can form in the legs or lungs after surgery, potentially causing pulmonary embolism.
9. Anesthesia-related complications: Respiratory and cardiac complications can occur during anesthesia, including respiratory and cardiac arrest.
10. delayed healing: Wound healing may be delayed or impaired after surgery, particularly in patients with pre-existing medical conditions.
It is important for patients to be aware of these potential complications and to discuss any concerns with their surgeon and healthcare team before undergoing surgery.
Ventral hernia is a type of hernia that occurs through a weakness in the abdominal wall, usually in the vicinity of the navel or groin. It can be caused by a variety of factors, including previous surgery, infection, or underlying weaknesses in the abdominal muscles.
The symptoms of ventral hernia may include a bulge or lump in the affected area, pain or discomfort, and difficulty with movement or exercise. If left untreated, ventral hernias can become larger and more difficult to repair, and may also lead to complications such as bowel obstruction or incarceration.
Treatment for ventral hernia usually involves surgical repair of the defect in the abdominal wall. The choice of surgical approach depends on the size and location of the hernia, as well as the patient's overall health and medical history. Open repair techniques involve a single incision in the abdomen to access the hernia sac and repair it with sutures or mesh material. Laparoscopic repair techniques involve several small incisions and the use of a camera and specialized instruments to repair the hernia through a minimally invasive approach.
In conclusion, ventral hernias are a common condition that can be repaired with a variety of surgical techniques. The choice of technique depends on the specific needs of the patient and the experience and expertise of the surgeon. Proper diagnosis and treatment are essential to prevent complications and ensure optimal outcomes for patients with ventral hernias.
Prevention of ventral hernia: the role of physical therapy. This article discusses the importance of physical therapy in preventing ventral hernias, particularly in high-risk populations such as those with previous abdominal surgery or chronic medical conditions. The authors review the scientific evidence supporting the use of physical therapy to improve abdominal strength and stability, reduce pressure on the abdominal wall, and promote healing after surgery.
The article highlights the benefits of preoperative physical therapy in preparing patients for surgery and reducing postoperative complications such as hernia recurrence or infection. Additionally, physical therapy can help patients recover more quickly and effectively after surgery, which may reduce hospital stays and improve overall outcomes.
The article also discusses the importance of individualized physical therapy programs tailored to each patient's specific needs and goals, as well as the role of technology such as biofeedback and electrical stimulation in enhancing physical therapy effectiveness.
In conclusion, physical therapy plays a critical role in preventing ventral hernias and promoting optimal outcomes for patients undergoing abdominal surgery. By improving abdominal strength and stability, reducing pressure on the abdominal wall, and promoting healing after surgery, physical therapy can help reduce the risk of complications and improve overall quality of life for high-risk patients.
The importance of ventral hernia repair in the elderly population: a review of the literature. This article examines the unique challenges and considerations associated with repairing ventral hernias in elderly patients. While hernia repair is generally considered safe and effective, elderly patients may be at higher risk for complications due to age-related changes such as decreased skin elasticity and muscle mass, comorbidities such as heart disease and diabetes, and potentially reduced physiological reserve.
The article highlights the importance of careful preoperative evaluation and planning, including a thorough medical history and physical examination, laboratory tests, and imaging studies to assess the severity of the hernia and potential for complications. Additionally, the use of advanced surgical techniques such as laparoscopic repair or robotic-assisted repair may be more challenging in elderly patients due to decreased visualization and dexterity, but can still provide excellent outcomes with careful planning and execution.
The article also discusses the importance of postoperative care in the elderly population, including close monitoring for signs of complications such as wound infections or respiratory failure, aggressive pain management to reduce the risk of respiratory depression, and early mobilization to promote healing and prevent delirium.
In conclusion, while repairing ventral hernias in elderly patients can be challenging, careful preoperative evaluation and planning, advanced surgical techniques, and meticulous postoperative care can provide excellent outcomes for this high-risk population.
There are several types of ischemia, including:
1. Myocardial ischemia: Reduced blood flow to the heart muscle, which can lead to chest pain or a heart attack.
2. Cerebral ischemia: Reduced blood flow to the brain, which can lead to stroke or cognitive impairment.
3. Peripheral arterial ischemia: Reduced blood flow to the legs and arms.
4. Renal ischemia: Reduced blood flow to the kidneys.
5. Hepatic ischemia: Reduced blood flow to the liver.
Ischemia can be diagnosed through a variety of tests, including electrocardiograms (ECGs), stress tests, and imaging studies such as CT or MRI scans. Treatment for ischemia depends on the underlying cause and may include medications, lifestyle changes, or surgical interventions.
There are several types of thrombosis, including:
1. Deep vein thrombosis (DVT): A clot forms in the deep veins of the legs, which can cause swelling, pain, and skin discoloration.
2. Pulmonary embolism (PE): A clot breaks loose from another location in the body and travels to the lungs, where it can cause shortness of breath, chest pain, and coughing up blood.
3. Cerebral thrombosis: A clot forms in the brain, which can cause stroke or mini-stroke symptoms such as weakness, numbness, or difficulty speaking.
4. Coronary thrombosis: A clot forms in the coronary arteries, which supply blood to the heart muscle, leading to a heart attack.
5. Renal thrombosis: A clot forms in the kidneys, which can cause kidney damage or failure.
The symptoms of thrombosis can vary depending on the location and size of the clot. Some common symptoms include:
1. Swelling or redness in the affected limb
2. Pain or tenderness in the affected area
3. Warmth or discoloration of the skin
4. Shortness of breath or chest pain if the clot has traveled to the lungs
5. Weakness, numbness, or difficulty speaking if the clot has formed in the brain
6. Rapid heart rate or irregular heartbeat
7. Feeling of anxiety or panic
Treatment for thrombosis usually involves medications to dissolve the clot and prevent new ones from forming. In some cases, surgery may be necessary to remove the clot or repair the damaged blood vessel. Prevention measures include maintaining a healthy weight, exercising regularly, avoiding long periods of immobility, and managing chronic conditions such as high blood pressure and diabetes.
The syndrome can be caused by a variety of factors, including:
* Compression from a tumor or other mass in the chest or neck
* Injury to the vein from trauma or surgery
* Blood clots or thrombophlebitis (inflammation of the vein wall)
* Infection or inflammation of the vein
* Cardiac tamponade (fluid accumulation in the pericardial sac surrounding the heart)
Symptoms of SVC syndrome can vary depending on the location and severity of the compression. They may include:
* Swelling of the face, neck, and arms
* Shortness of breath
* Difficulty speaking or swallowing
* Pain in the head, neck, or chest
* Fatigue or weakness
* Decreased consciousness or confusion
If you suspect that you or someone else may be experiencing SVC syndrome, it is important to seek medical attention immediately. A healthcare provider will perform a physical examination and order diagnostic tests, such as imaging studies or blood tests, to determine the cause of the symptoms and develop an appropriate treatment plan.
Treatment for SVC syndrome may include:
* Anticoagulation medications to prevent blood clots from forming
* Pain management medications to relieve swelling and discomfort
* Surgery to remove a tumor or other mass compressing the vein
* Endovascular procedures, such as angioplasty or stenting, to open up the vein and restore blood flow
* Supportive care, such as oxygen therapy or mechanical ventilation, in severe cases.
Early diagnosis and treatment are critical to prevent complications and improve outcomes for patients with SVC syndrome. If you suspect that you or someone else may be experiencing symptoms of this condition, do not hesitate to seek medical attention right away.
There are several types of aneurysms, including:
1. Thoracic aneurysm: This type of aneurysm occurs in the chest cavity and is usually caused by atherosclerosis or other conditions that affect the aorta.
2. Abdominal aneurysm: This type of aneurysm occurs in the abdomen and is usually caused by high blood pressure or atherosclerosis.
3. Cerebral aneurysm: This type of aneurysm occurs in the brain and can cause symptoms such as headaches, seizures, and stroke.
4. Peripheral aneurysm: This type of aneurysm occurs in the peripheral arteries, which are the blood vessels that carry blood to the arms and legs.
Symptoms of an aneurysm can include:
1. Pain or discomfort in the affected area
2. Swelling or bulging of the affected area
3. Weakness or numbness in the affected limb
4. Shortness of breath or chest pain (in the case of a thoracic aneurysm)
5. Headaches, seizures, or stroke (in the case of a cerebral aneurysm)
If an aneurysm is not treated, it can lead to serious complications such as:
1. Rupture: This is the most serious complication of an aneurysm and occurs when the aneurysm sac bursts, leading to severe bleeding and potentially life-threatening consequences.
2. Stroke or brain damage: If a cerebral aneurysm ruptures, it can cause a stroke or brain damage.
3. Infection: An aneurysm can become infected, which can lead to serious health problems.
4. Blood clots: An aneurysm can form blood clots, which can break loose and travel to other parts of the body, causing blockages or further complications.
5. Kidney failure: If an aneurysm is not treated, it can cause kidney failure due to the pressure on the renal arteries.
6. Heart problems: An aneurysm in the aorta can lead to heart problems such as heart failure or cardiac arrest.
7. Sepsis: If an aneurysm becomes infected, it can lead to sepsis, which is a life-threatening condition that can cause organ failure and death.
Treatment options for an aneurysm include:
1. Observation: Small aneurysms that are not causing any symptoms may not require immediate treatment and can be monitored with regular check-ups to see if they are growing or changing.
2. Surgery: Open surgery or endovascular repair are two common methods for treating aneurysms. In open surgery, the surgeon makes an incision in the abdomen to repair the aneurysm. In endovascular repair, a small tube is inserted into the affected blood vessel through an incision in the groin, and then guided to the site of the aneurysm where it is expanded to fill the aneurysm sac and seal off the aneurysm.
3. Embolization: This is a minimally invasive procedure where a small catheter is inserted into the affected blood vessel through an incision in the groin, and then guided to the site of the aneurysm where it releases tiny particles or coils that fill the aneurysm sac and seal off the aneurysm.
4. Medications: Certain medications such as antibiotics and blood thinners may be prescribed to treat related complications such as infection or blood clots.
It is important to seek medical attention if you experience any symptoms of an aneurysm, such as sudden severe headache, vision changes, difficulty speaking, weakness or numbness in the face or limbs, as prompt treatment can help prevent complications and improve outcomes.
Some examples of pathologic constrictions include:
1. Stenosis: A narrowing or constriction of a blood vessel or other tubular structure, often caused by the buildup of plaque or scar tissue.
2. Asthma: A condition characterized by inflammation and constriction of the airways, which can make breathing difficult.
3. Esophageal stricture: A narrowing of the esophagus that can cause difficulty swallowing.
4. Gastric ring constriction: A narrowing of the stomach caused by a band of tissue that forms in the upper part of the stomach.
5. Anal fissure: A tear in the lining of the anus that can cause pain and difficulty passing stools.
Pathologic constrictions can be caused by a variety of factors, including inflammation, infection, injury, or genetic disorders. They can be diagnosed through imaging tests such as X-rays, CT scans, or endoscopies, and may require surgical treatment to relieve symptoms and improve function.
Example sentences:
1. The patient developed a foreign-body reaction after receiving a defective hip implant, resulting in severe pain and swelling.
2. The transplanted liver was rejected by the recipient's immune system, causing a foreign-body reaction that led to its failure.
3. The use of a certain drug was associated with a high risk of foreign-body reactions, leading to its withdrawal from the market.
It is important to identify and address prosthesis failure early to prevent further complications and restore the functionality of the device. This may involve repairing or replacing the device, modifying the design, or changing the materials used in its construction. In some cases, surgical intervention may be necessary to correct issues related to the implantation of the prosthetic device.
Prosthesis failure can occur in various types of prosthetic devices, including joint replacements, dental implants, and orthotic devices. The causes of prosthesis failure can range from manufacturing defects to user error or improper maintenance. It is essential to have a comprehensive understanding of the factors contributing to prosthesis failure to develop effective solutions and improve patient outcomes.
In conclusion, prosthesis failure is a common issue that can significantly impact the quality of life of individuals who rely on prosthetic devices. Early identification and addressing of prosthesis failure are crucial to prevent further complications and restore functionality. A comprehensive understanding of the causes of prosthesis failure is necessary to develop effective solutions and improve patient outcomes.
Types of Scleral Diseases:
1. Scleritis: This is an inflammatory condition that affects the sclera, causing redness, pain, and swelling. It can be caused by infection, autoimmune disorders, or other factors.
2. Sclerochoroidal detachment: This is a condition where the sclera pulls away from the choroid, a layer of blood vessels beneath the retina. It can cause vision loss and is often seen in patients with uveitis or other inflammatory conditions.
3. Scleral rupture: This is a rare condition where the sclera tears or ruptures, causing sudden vision loss. It is often caused by trauma or inflammation.
4. Scleroconjunctivitis: This is an inflammatory condition that affects the sclera and conjunctiva, causing redness, discharge, and sensitivity to light.
5. Sclerodermia: This is a rare genetic disorder that affects the skin and eyes, including the sclera. It can cause thinning of the skin and abnormal growth of tissue.
Symptoms of Scleral Diseases:
The symptoms of scleral diseases can vary depending on the specific condition, but may include:
* Redness and inflammation in the eye
* Pain or discomfort in the eye
* Blurred vision or vision loss
* Sensitivity to light
* Discharge or tearing
* Swelling of the eye
* Bulging of the eye
* Abnormal growth of tissue
Diagnosis and Treatment of Scleral Diseases:
Diagnosis of scleral diseases is typically made through a combination of physical examination, imaging tests such as ultrasound or MRI, and laboratory tests to rule out other conditions. Treatment depends on the specific condition and may include:
* Medications such as anti-inflammatory drugs or antibiotics
* Surgery to remove abnormal tissue or correct structural problems
* Laser therapy to improve vision or reduce inflammation
* Injections of medication into the eye
* Oral medications to treat underlying conditions such as autoimmune disorders or infections.
Prognosis:
The prognosis for scleral diseases varies depending on the specific condition and the severity of the symptoms. In general, early diagnosis and treatment can improve the outlook for many of these conditions. However, some scleral diseases may have a poor prognosis if left untreated or if they are not properly managed. It is important to seek medical attention if you experience any symptoms of scleral disease.
Prevention:
While some scleral diseases may be genetic and cannot be prevented, there are steps you can take to reduce your risk of developing certain conditions. For example:
* Avoid exposure to harmful substances such as chemicals or radiation
* Wear protective eyewear when performing activities that could potentially damage the eyes
* Maintain a healthy diet and exercise regularly to reduce your risk of developing underlying conditions such as diabetes or high blood pressure.
It is important to note that some scleral diseases may not have any noticeable symptoms until they are advanced, so it is important to receive regular eye exams to detect any potential issues early on.
In conclusion, scleral diseases can cause a wide range of symptoms and can be challenging to diagnose and treat. However, with proper medical attention and self-care, many people with scleral diseases are able to manage their symptoms and improve their quality of life. It is important to seek medical attention if you experience any symptoms of scleral disease, and to maintain regular eye exams to detect any potential issues early on.
Eventration is most commonly observed on the left side and can be diagnosed using imaging techniques such as chest X-rays or CT scans. Symptoms may include respiratory distress, gastroesophageal reflux, and difficulty swallowing. Treatment options vary depending on the severity of the condition and may involve surgical intervention to repair any associated hernias or other structural anomalies. In some cases, eventration may be an incidental finding during unrelated medical testing.
The term "diaphragmatic eventration" is used to describe this condition specifically when it affects the diaphragm. The diaphragm is a dome-shaped muscle that separates the chest cavity from the abdominal cavity and plays a crucial role in breathing. Eventration of the diaphragm can disrupt normal breathing function and may lead to complications such as respiratory failure or gastrointestinal obstruction if left untreated.
In summary, diaphragmatic eventration is a rare congenital condition characterized by abnormal dilatation or invagination of the diaphragm, which may be associated with other structural anomalies. It can cause respiratory and gastrointestinal symptoms and may require surgical intervention for proper treatment and management.
Types of vascular neoplasms include:
1. Hemangiomas: These are benign tumors that arise from abnormal blood vessels and are most common in infants and children.
2. Lymphangiomas: These are benign tumors that arise from the lymphatic system and are also more common in children.
3. Vasculitis: This is a condition where the blood vessels become inflamed, leading to the formation of tumors.
4. Angiosarcoma: This is a rare and malignant tumor that arises from the blood vessels.
5. Lymphangioendotheliomas: These are rare benign tumors that arise from the lymphatic system.
Symptoms of vascular neoplasms can vary depending on their location and size, but may include:
* Pain or discomfort in the affected area
* Swelling or bruising
* Redness or warmth in the skin
* Difficulty moving or bending
Diagnosis of vascular neoplasms typically involves a combination of imaging tests such as ultrasound, CT scans, and MRI, along with a biopsy to confirm the presence of cancer cells. Treatment options depend on the type and location of the tumor, but may include surgery, chemotherapy, or radiation therapy.
It is important to seek medical attention if you experience any persistent symptoms or notice any unusual changes in your body, as early diagnosis and treatment can improve outcomes for vascular neoplasms.
There are two main types of carotid stenosis:
1. Internal carotid artery stenosis: This type of stenosis occurs when the internal carotid artery, which supplies blood to the brain, becomes narrowed or blocked.
2. Common carotid artery stenosis: This type of stenosis occurs when the common carotid artery, which supplies blood to the head and neck, becomes narrowed or blocked.
The symptoms of carotid stenosis can vary depending on the severity of the blockage and the extent of the affected area. Some common symptoms include:
* Dizziness or lightheadedness
* Vertigo (a feeling of spinning)
* Blurred vision or double vision
* Memory loss or confusion
* Slurred speech
* Weakness or numbness in the face, arm, or leg on one side of the body
If left untreated, carotid stenosis can lead to a stroke or other serious complications. Treatment options for carotid stenosis include medications to lower cholesterol and blood pressure, as well as surgical procedures such as endarterectomy (removing plaque from the artery) or stenting (placing a small mesh tube in the artery to keep it open).
In conclusion, carotid stenosis is a serious medical condition that can lead to stroke and other complications if left untreated. It is important to seek medical attention if symptoms persist or worsen over time.
An abdominal aortic aneurysm can cause symptoms such as abdominal pain, back pain, and difficulty breathing if it ruptures. It can also be diagnosed through imaging tests such as ultrasound, CT scan, or MRI. Treatment options for an abdominal aortic aneurysm include watchful waiting (monitoring the aneurysm for signs of growth or rupture), endovascular repair (using a catheter to repair the aneurysm from within the blood vessel), or surgical repair (open surgery to repair the aneurysm).
Word Origin and History
The word 'aneurysm' comes from the Greek words 'aneurysma', meaning 'dilation' and 'sma', meaning 'a vessel'. The term 'abdominal aortic aneurysm' was first used in the medical literature in the late 19th century to describe this specific type of aneurysm.
Prevalence and Incidence
Abdominal aortic aneurysms are relatively common, especially among older adults. According to the Society for Vascular Surgery, approximately 2% of people over the age of 65 have an abdominal aortic aneurysm. The prevalence of abdominal aortic aneurysms increases with age, and men are more likely to be affected than women.
Risk Factors
Several risk factors can increase the likelihood of developing an abdominal aortic aneurysm, including:
* High blood pressure
* Atherosclerosis (hardening of the arteries)
* Smoking
* Family history of aneurysms
* Previous heart attack or stroke
* Marfan syndrome or other connective tissue disorders.
Symptoms and Diagnosis
Abdominal aortic aneurysms can be asymptomatic, meaning they do not cause any noticeable symptoms. However, some people may experience symptoms such as:
* Abdominal pain or discomfort
* Back pain
* Weakness or fatigue
* Palpitations
* Shortness of breath
If an abdominal aortic aneurysm is suspected, several diagnostic tests may be ordered, including:
* Ultrasound
* Computed tomography (CT) scan
* Magnetic resonance imaging (MRI)
* Angiography
Treatment and Management
The treatment of choice for an abdominal aortic aneurysm depends on several factors, including the size and location of the aneurysm, as well as the patient's overall health. Treatment options may include:
* Watchful waiting (for small aneurysms that are not causing any symptoms)
* Endovascular repair (using a stent or other device to repair the aneurysm from within the blood vessel)
* Open surgical repair (where the surgeon makes an incision in the abdomen to repair the aneurysm)
In some cases, emergency surgery may be necessary if the aneurysm ruptures or shows signs of impending rupture.
Complications and Risks
Abdominal aortic aneurysms can lead to several complications and risks, including:
* Rupture (which can be life-threatening)
* Infection
* Blood clots or blockages in the blood vessels
* Kidney damage
* Heart problems
Prevention
There is no guaranteed way to prevent an abdominal aortic aneurysm, but several factors may reduce the risk of developing one. These include:
* Maintaining a healthy lifestyle (including a balanced diet and regular exercise)
* Not smoking
* Managing high blood pressure and other medical conditions
* Getting regular check-ups with your healthcare provider
Prognosis and Life Expectancy
The prognosis for abdominal aortic aneurysms depends on several factors, including the size of the aneurysm, its location, and whether it has ruptured. In general, the larger the aneurysm, the poorer the prognosis. If treated before rupture, many people with abdominal aortic aneurysms can expect a good outcome and a normal life expectancy. However, if the aneurysm ruptures, the survival rate is much lower.
In conclusion, abdominal aortic aneurysms are a serious medical condition that can be life-threatening if left untreated. It is important to be aware of the risk factors and symptoms of an aneurysm, and to seek medical attention immediately if any are present. With proper treatment, many people with abdominal aortic aneurysms can expect a good outcome and a normal life expectancy.
Polytetrafluoroethylene
Surgical suture
Gore-Tex
Perfluorinated compound
Timeline of events related to per- and polyfluoroalkyl substances
Rachel Makinson
Pulmonary artery banding
Kristy M. Ainslie
Fluorographene
Young's modulus
Surface treatment of PTFE
Francis Rudolph Shonka
Hydrogen pipeline transport
Barbara S. Larsen
Gas chromatography-olfactometry
Organofluorine chemistry
Femoropopliteal bypass
Boron trifluoride
Hessam Nowzari
Arcjet rocket
Rhinoplasty
Baden-Württemberg 1
Roy J. Plunkett
Ion track
German space programme
Electroless nickel-phosphorus plating
1938 in science
April 1938
Materials for use in vacuum
Oral hygiene
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PTFE10
- This study proposes that a polytetrafluoroethylene ( PTFE ) electret tube charged by frictional electricity can prevent the solidification of the indwelling catheter in blood vessels . (bvsalud.org)
- After extraction of the maxillary right incisor, the animals of the test groups were subjected to alveolar guided bone regeneration (GBR) surgery and received an expanded polytetrafluoroethylene (e-PTFE) and a latex membrane, respectively. (bvsalud.org)
- The Polytetrafluoroethylene (PTFE) Market research study provides a detailed survey of the vital players - this is based on the financial highlights, company outline, SWOT Analysis, Product Portfolio, as well as major strategies and the expansion plans of industry contenders. (cmferesearch.com)
- The competitive analysis comprising numerous market players is a rather noteworthy feature of the Polytetrafluoroethylene (PTFE) market report, as it provides details about the direct and indirect competitors in the market. (cmferesearch.com)
- Also, the report compares the growth rate and production value of the Polytetrafluoroethylene (PTFE) market spanning different geographies. (cmferesearch.com)
- In this study, we analyzed the early stages of bacterial adhesion on two commercial dense polytetrafluoroethylene (d-PTFE) membranes in order to identify microstructural features that led to different adhesion strengths. (nih.gov)
- Airborne nanoparticle concentrations in the manufacturing of polytetrafluoroethylene (PTFE) apparel. (cdc.gov)
- One form of waterproof, breathable apparel is manufactured from polytetrafluoroethylene (PTFE) membrane laminated fabric using a specific process to seal seams that have been sewn with traditional techniques. (cdc.gov)
- A strip-shield is a coil liner consisting of thin copper strips layered on a PTFE (polytetrafluoroethylene) insulator. (nih.gov)
- Perfluorooctanoic acid (PFOA) is used primarily to produce salts which are used in the production of fluoroelastomers and fluoropolymers, such as polytetrafluoroethylene (PTFE) and polyvinylidine fluoride (PVDF). (cdc.gov)
Teflon1
- Tetrafluoroethylene is used in the production of polytetrafluoroethylene (Teflon®) and other polymers. (nih.gov)
Fluoropolymer1
- Polytetrafluoroethylene is a synthetic fluoropolymer of tetrafluoroethylene that has numerous applications. (goodfellow.com)
Fluoride1
- 3. Its chemical resistance is similar to that of polytetrafluoroethylene and better than that of vinylidene fluoride. (tianswax.com)
Lead1
- For example, inhalation of thermal degradation products of polytetrafluoroethylene can lead to "polymer fume fever" [10] and in extreme cases to fatal acute pulmonary oedema [11]. (cdc.gov)
China1
- On August 23, 2021, DGTR issued final findings recommending continuation of anti-dumping duty on import of Melamine from China and Polytetrafluoroethylene from Russia. (nityatax.com)
Material1
- Polytetrafluoroethylene Fiberglass Bush has a cylindrical shape and material used in its processing is glass filled polytetrafluoroethylene. (sanghvitechnoproducts.co.in)
Grafts5
- Release of PDGF-BB and bFGF by human endothelial cells seeded on expanded polytetrafluoroethylene vascular grafts. (bvsalud.org)
- 6. A contemporary meta-analysis of Dacron versus polytetrafluoroethylene grafts for femoropopliteal bypass grafting. (nih.gov)
- 7. Heparin-bonded expanded polytetrafluoroethylene femoropopliteal bypass grafts outperform expanded polytetrafluoroethylene grafts without heparin in a long-term comparison. (nih.gov)
- 16. [Dacron and polytetrafluoroethylene aorto-bifemoral grafts]. (nih.gov)
- 19. Midterm Results of a Japanese Prospective Multicenter Registry of Heparin-Bonded Expanded Polytetrafluoroethylene Grafts for Above-the-Knee Femoropopliteal Bypass. (nih.gov)
Vascular1
- 3. Randomized controlled trial comparing the safety and efficacy between the FUSION BIOLINE heparin-coated vascular graft and the standard expanded polytetrafluoroethylene graft for femoropopliteal bypass. (nih.gov)
Substance1
- It contains a substance called polytetrafluoroethylene. (medlineplus.gov)