Peroneal Nerve
Peroneal Neuropathies
Tibial Nerve
Sural Nerve
Peripheral Nerves
Sciatic Nerve
Foot
Nerve Compression Syndromes
Neural Conduction
Nerve Fibers
Sympathetic Nervous System
Paralysis
Peripheral Nervous System Diseases
Optic Nerve
Nerve Block
Implantable Neurostimulators
Median Nerve
Electromyography
Femoral Nerve
Nerve Transfer
Gait Disorders, Neurologic
Electrodiagnosis
H-Reflex
Fibula
Ankle Joint
Tendon Transfer
Muscle, Skeletal
Nerve Endings
Facial Nerve
Transcutaneous Electric Nerve Stimulation
Ulnar Nerve
Wavelet Analysis
Reflex
Spinal Nerves
Dissection
Diabetic Neuropathies
Nerve Growth Factor
Trigeminal Nerve
Nerve Growth Factors
Skin
Phrenic Nerve
Muscle Spindles
Radial Nerve
Muscle Contraction
Cranial Nerves
Spinal Nerve Roots
Posterior Cruciate Ligament
Action Potentials
Orthotic Devices
Ophthalmic Nerve
Patient Positioning
Afferent Pathways
Hindlimb
Nerve Tissue
Recovery of Function
Mandibular Nerve
Mechanoreceptors
Paresis
Cats
Sweating
Specific and innervation-regulated expression of the intermediate filament protein nestin at neuromuscular and myotendinous junctions in skeletal muscle. (1/476)
The intermediate filament proteins nestin, vimentin, and desmin show a specific temporal expression pattern during the development of myofibers from myogenic precursor cells. Nestin and vimentin are actively expressed during early developmental stages to be later down-regulated, vimentin completely and nestin to minimal levels, whereas desmin expression begins later and is maintained in mature myofibers, in which desmin participates in maintaining structural integrity. In this study we have analyzed the expression levels and distribution pattern of nestin in intact and denervated muscle in rat and in human. Nestin immunoreactivity was specifically and focally localized in the sarcoplasm underneath neuromuscular junctions (NMJs) and in the vicinity of the myotendinous junctions (MTJs), ie, in regions associated with acetylcholine receptors (AChRs). This association prompted us to analyze nestin in neurogenically and myogenically denervated muscle. Immunoblot analysis disclosed a marked overall increase of accumulated nestin protein. Similar to the extrajunctional redistribution of AChRs in denervated myofibers, nestin immunoreactivity extended widely beyond the NMJ region. Re-innervation caused complete reversion of these changes. Our study demonstrates that the expression levels and distribution pattern of nestin are regulated by innervation, ie, signal transduction into myofibers. (+info)Uninjured C-fiber nociceptors develop spontaneous activity and alpha-adrenergic sensitivity following L6 spinal nerve ligation in monkey. (2/476)
We investigated whether uninjured cutaneous C-fiber nociceptors in primates develop abnormal responses after partial denervation of the skin. Partial denervation was induced by tightly ligating spinal nerve L6 that innervates the dorsum of the foot. Using an in vitro skin-nerve preparation, we recorded from uninjured single afferent nerve fibers in the superficial peroneal nerve. Recordings were made from 32 C-fiber nociceptors 2-3 wk after ligation and from 29 C-fiber nociceptors in control animals. Phenylephrine, a selective alpha1-adrenergic agonist, and UK14304 (UK), a selective alpha2-adrenergic agonist, were applied to the receptive field for 5 min in increasing concentrations from 0.1 to 100 microM. Nociceptors from in vitro control experiments were not significantly different from nociceptors recorded by us previously in in vivo experiments. In comparison to in vitro control animals, the afferents found in lesioned animals had 1) a significantly higher incidence of spontaneous activity, 2) a significantly higher incidence of response to phenylephrine, and 3) a higher incidence of response to UK. In lesioned animals, the peak response to phenylephrine was significantly greater than to UK, and the mechanical threshold of phenylephrine-sensitive afferents was significantly lower than for phenylephrine-insensitive afferents. Staining with protein gene product 9.5 revealed an approximately 55% reduction in the number of unmyelinated terminals in the epidermis of the lesioned limb compared with the contralateral limb. Thus uninjured cutaneous C-fiber nociceptors that innervate skin partially denervated by ligation of a spinal nerve acquire two abnormal properties: spontaneous activity and alpha-adrenergic sensitivity. These abnormalities in nociceptor function may contribute to neuropathic pain. (+info)Arousal from sleep shortens sympathetic burst latency in humans. (3/476)
1. Bursts of sympathetic activity in muscle nerves are phase-locked to the cardiac cycle by the sinoaortic baroreflexes. Acoustic arousal from non-rapid eye movement (NREM) sleep reduces the normally invariant interval between the R-wave of the electrocardiogram (ECG) and the peak of the corresponding sympathetic burst; however, the effects of other forms of sleep disruption (i.e. spontaneous arousals and apnoea-induced arousals) on this temporal relationship are unknown. 2. We simultaneously recorded muscle sympathetic nerve activity in the peroneal nerve (intraneural electrodes) and the ECG (surface electrodes) in seven healthy humans and three patients with sleep apnoea syndrome during NREM sleep. 3. In seven subjects, burst latencies were shortened subsequent to spontaneous K complexes (1.297 +/- 0.024 s, mean +/- s. e.m.) and spontaneous arousals (1.268 +/- 0.044 s) compared with latencies during periods of stable NREM sleep (1.369 +/- 0.023 s). In six subjects who demonstrated spontaneous apnoeas during sleep, apnoea per se did not alter burst latency relative to sleep with stable electroencephalogram (EEG) and breathing (1.313 +/- 0.038 vs. 1.342 +/- 0.026 s); however, following apnoea-induced EEG perturbations, burst latencies were reduced (1.214 +/- 0.034 s). 4. Arousal-induced reduction in sympathetic burst latency may reflect a temporary diminution of baroreflex buffering of sympathetic outflow. If so, the magnitude of arterial pressure perturbations during sleep (e.g. those caused by sleep disordered breathing and periodic leg movements) may be augmented by arousal. (+info)Activity-dependent slowing of conduction differentiates functional subtypes of C fibres innervating human skin. (4/476)
1. The effects of impulse activity on conduction in cutaneous C fibres have been examined in 46 microneurographic recordings from 11 normal subjects and 11 diabetic patients with normal nerve conduction. A tungsten microelectrode was inserted into a cutaneous nerve, usually the superficial peroneal close to the ankle, and intraneural microstimulation was used to identify an area of skin innervated. Three minute trains of 0.25 ms stimuli at 1, 2 and 4 Hz were then delivered to the surface of the skin, separated by intervals of 6 min with stimulation at 0.25 Hz. Slowing and block of conduction were measured from the nerve responses for up to seven C units per stimulation sequence. 2. Three types of C unit were distinguished by their responses to repetitive stimulation: type 1 units slowed progressively during the 3 min trains; slowing of type 2 units reached a plateau within 1 min; while type 3 units hardly slowed at all. Data from normal and diabetic subjects did not differ and were pooled. After 3 min at 2 Hz, the percentage increases in latency were for type 1, 28.3 +/- 9.7 (n = 63 units, mean +/- s.d.); for type 2, 5.2 +/- 1.6 (n = 14); and for type 3, 0.8 +/- 0.5 (n = 5), with no overlap. After 3 min at 4 Hz, 58 % of type 1 units (but no type 2 or 3 units) blocked intermittently. Recovery of latency after stimulation was faster for type 2 than for type 1 units, but conduction velocities of the three types were similar. 3. Type 1 units were identified as nociceptors and 7 type 2 units were identified as 'cold' fibres, activated by non-noxious cold, with no overlap in modality. None of the units tested was activated by weak mechanical stimuli or reflex sympathetic activation. 4. Spike waveforms were averaged for 18 type 1, 10 type 2 and 6 type 3 units. All units had predominantly triphasic action potentials with a major negative peak, but those of type 3 units were on average both smaller and briefer than those of type 1 and type 2 units. 5. It is concluded that repetitive electrical stimulation reliably differentiates nociceptive from cold-specific C fibres innervating human hairy skin, as has previously been shown for the rat. Cold fibres can propagate impulses continuously at much higher rates than nociceptive fibres. The nature of the type 3 units is unclear. (+info)Respiratory and cardiac modulation of single sympathetic vasoconstrictor and sudomotor neurones to human skin. (5/476)
1. The firing of single sympathetic neurones was recorded via tungsten microelectrodes in cutaneous fascicles of the peroneal nerve in awake humans. Studies were made of 17 vasoconstrictor neurones during cold-induced cutaneous vasoconstriction and eight sudomotor neurones during heat-induced sweating. Oligounitary recordings were obtained from 8 cutaneous vasconstrictor and 10 sudomotor sites. Skin blood flow was measured by laser Doppler flowmetry, and sweating by changes in skin electrical resistance within the innervation territory on the dorsum of the foot. 2. Perispike time histograms revealed respiratory modulation in 11 (65 %) vasoconstrictor and 4 (50 %) sudomotor neurones. After correcting for estimated conduction delays, the firing probability was higher in inspiration for both classes of neurone. Measured from the oligounitary recordings, the respiratory modulation indices were 67. 7 +/- 3.9 % for vasoconstrictor and 73.5 +/- 5.7 % for sudomotor neurones (means +/- s.e.m.). As previously found for sudomotor neurones, cardiac rhythmicity was expressed by 7 (41 %) vasoconstrictor neurones, 5 of which showed no significant coupling to respiration. Measured from the oligounitary records, the cardiac modulation of cutaneous vasoconstrictor activity was 58.6 +/- 4.9 %, compared with 74.4 +/- 6.4 % for sudomotor activity. 3. Both vasoconstrictor and sudomotor neurones displayed low average firing rates (0.53 and 0.62 Hz, respectively). The percentage of cardiac intervals in which units fired was 38 % and 35 %, respectively. Moreover, when considering only those cardiac intervals when a unit fired, vasoconstrictor and sudomotor neurones generated a single spike 66 % and 67 % of the time. Rarely were more than four spikes generated by a single neurone. 4. We conclude that human cutaneous vasoconstrictor and sudomotor neurones share several properties: both classes contain subpopulations that are modulated by respiration and/or the cardiac cycle. The data suggest that the intensity of a multi-unit burst of vasoconstrictor or sudomotor impulses is probably governed primarily by firing incidence and the recruitment of additional neurones, rather than by an increase in the number of spikes each unit contributes to a burst. (+info)Responses of sympathetic outflow to skin during caloric stimulation in humans. (6/476)
We previously showed that caloric vestibular stimulation elicits increases in sympathetic outflow to muscle (MSNA) in humans. The present study was conducted to determine the effect of this stimulation on sympathetic outflow to skin (SSNA). The SSNA in the tibial and peroneal nerves and nystagmus was recorded in nine subjects when the external meatus was irrigated with 50 ml of cold (10 degrees C) or warm (44 degrees C) water. During nystagmus, the SSNA in tibial and peroneal nerves decreased to 50 +/- 4% (with baseline value set as 100%) and 61 +/- 4%, respectively. The degree of SSNA suppression in both nerves was proportional to the maximum slow-phase velocity of nystagmus. After nystagmus, the SSNA increased to 166 +/- 7 and 168 +/- 6%, respectively, and the degree of motion sickness symptoms was correlated with this SSNA increase. These results suggest that the SSNA response differs from the MSNA response during caloric vestibular stimulation and that the SSNA response elicited in the initial period of caloric vestibular stimulation is different from that observed during the period of motion sickness symptoms. (+info)Induction of neurally mediated syncope with adenosine. (7/476)
BACKGROUND: Tilt testing is used to establish the diagnosis of neurally mediated syncope. However, applicability of the tilt test is limited by test sensitivity and length of time required to perform the test. We hypothesized that adenosine could facilitate the induction of neurally mediated syncope through its sympathomimetic effects and therefore could be used as an alternative to routine tilt testing. METHODS AND RESULTS: In protocol 1, the yield of adenosine tilt testing (12 mg while upright, followed by 60 degrees tilt for 5 minutes) and a 15-minute isoproterenol tilt test were compared in 84 patients with a negative 30-minute drug-free tilt test. In protocol 2, 100 patients underwent an initial adenosine tilt test followed by our routine tilt test (30-minute drug-free tilt followed by a 15-minute isoproterenol tilt). Six additional control patients underwent microneurography of the peroneal nerve to compare the sympathomimetic effects during bolus administration of adenosine and continuous infusion of isoproterenol. In protocol 1, the yields of adenosine (8 of 84, 10%) and isoproterenol (7 of 84, 8%) tilt testing were comparable (P=NS). In protocol 2, the yields of adenosine (19 of 100, 19%) and routine (22 of 100, 22%) tilt testing were also comparable (P=NS). Although the yield of adenosine tilt testing was comparable in both protocols, patients with a negative adenosine tilt test but a positive routine tilt test usually required isoproterenol to elicit the positive response. Microneurography confirmed discordant sympathetic activation after adenosine and isoproterenol administration. CONCLUSIONS: Adenosine is effective for the induction of neurally mediated syncope, with a diagnostic yield comparable to routine tilt testing. However, the discordant results obtained with adenosine and the isoproterenol phase of routine tilt testing suggest that adenosine and isoproterenol tilt testing may have complementary roles in eliciting a positive response. Therefore, a tilt protocol that uses an initial adenosine tilt followed, if necessary, by an isoproterenol tilt would be expected to increase the overall yield and reduce the duration of tilt testing. (+info)Aberrant neurofilament phosphorylation in sensory neurons of rats with diabetic neuropathy. (8/476)
Aberrant neurofilament phosphorylation occurs in many neurodegenerative diseases, and in this study, two animal models of type 1 diabetes--the spontaneously diabetic BB rat and the streptozocin-induced diabetic rat--have been used to determine whether such a phenomenon is involved in the etiology of the symmetrical sensory polyneuropathy commonly associated with diabetes. There was a two- to threefold (P < 0.05) elevation of neurofilament phosphorylation in lumbar dorsal root ganglia (DRG) of diabetic rats that was localized to perikarya of medium to large neurons using immunocytochemistry. Additionally, diabetes enhanced neurofilament M phosphorylation by 2.5-fold (P < 0.001) in sural nerve of BB rats. Neurofilaments are substrates of the mitogen-activated protein kinase (MAPK) family, which includes c-jun NH2-terminal kinase (JNK) or stress-activated protein kinase (SAPK1) and extracellular signal-regulated kinases (ERKs) 1 and 2. Diabetes induced a significant three- to fourfold (P < 0.05) increase in phosphorylation of a 54-kDa isoform of JNK in DRG and sural nerve, and this correlated with elevated c-Jun and neurofilament phosphorylation. In diabetes, ERK phosphorylation was also increased in the DRG, but not in sural nerve. Immunocytochemistry showed that JNK was present in sensory neuron perikarya and axons. Motoneuron perikarya and peroneal nerve of diabetic rats showed no evidence of increased neurofilament phosphorylation and failed to exhibit phosphorylation of JNK. It is hypothesized that in sensory neurons of diabetic rats, aberrant phosphorylation of neurofilament may contribute to the distal sensory axonopathy observed in diabetes. (+info)Peroneal neuropathies are a group of disorders that affect the nerves in the peroneal region of the leg. The peroneal nerves are responsible for controlling the muscles in the calf and foot, and when they are damaged or diseased, it can cause a range of symptoms, including weakness, numbness, tingling, and pain in the affected area. There are several different types of peroneal neuropathies, including: 1. Chronic compression neuropathy: This occurs when the peroneal nerve is compressed or pinched, often due to a herniated disk in the lower back or a tumor in the leg. 2. Inflammatory neuropathy: This is caused by an autoimmune response that attacks the nerve, leading to inflammation and damage. 3. Idiopathic neuropathy: This type of peroneal neuropathy has no known cause and is often referred to as "idiopathic" or "unknown." 4. Hereditary neuropathy: There are several genetic disorders that can cause peroneal neuropathy, including Charcot-Marie-Tooth disease and hereditary neuropathy with liability to pressure palsies. Treatment for peroneal neuropathies depends on the underlying cause and the severity of the symptoms. In some cases, conservative treatments such as physical therapy, pain management, and lifestyle changes may be sufficient. In more severe cases, surgery may be necessary to relieve pressure on the nerve or to repair damage to the nerve.
Knee dislocation is a medical condition in which the knee joint is completely out of its normal position. This can occur when the bones of the knee joint are forced apart, either due to a direct blow or a twisting injury. Knee dislocations are typically classified into two types: anterior dislocations and posterior dislocations. An anterior dislocation occurs when the lower leg bone (tibia) is forced forward and out of its normal position in relation to the thigh bone (femur). A posterior dislocation occurs when the lower leg bone is forced backward and out of its normal position in relation to the thigh bone. Knee dislocations can be very painful and can cause significant damage to the surrounding ligaments, tendons, and muscles. Treatment typically involves surgery to realign the bones and repair any damaged tissues. Physical therapy may also be necessary to help restore strength and range of motion in the knee joint.,。
Nerve compression syndromes are a group of conditions that occur when a nerve is compressed or pinched, leading to pain, numbness, weakness, or other symptoms. These conditions can affect any nerve in the body, but are most commonly seen in the neck, back, and extremities. There are several types of nerve compression syndromes, including carpal tunnel syndrome, cubital tunnel syndrome, radial tunnel syndrome, tarsal tunnel syndrome, and sciatica. These conditions can be caused by a variety of factors, including repetitive motions, poor posture, injury, or underlying medical conditions such as arthritis or diabetes. Treatment for nerve compression syndromes typically involves addressing the underlying cause of the compression, such as through physical therapy, medication, or surgery. In some cases, lifestyle changes such as improving posture or modifying work habits may also be recommended to prevent further compression of the affected nerve.
In the medical field, paralysis refers to a loss of muscle function or weakness in one or more areas of the body. This can be caused by a variety of factors, including injury, disease, or neurological disorders. There are several types of paralysis, including: 1. Complete paralysis: This is when a person is unable to move any part of their body. 2. Partial paralysis: This is when a person has some muscle function, but not all of it. 3. Flaccid paralysis: This is when the muscles are weak and floppy, and the person may have difficulty moving or maintaining their posture. 4. Spastic paralysis: This is when the muscles are tight and tense, and the person may have difficulty controlling their movements. Paralysis can affect any part of the body, including the arms, legs, face, and voice. It can be temporary or permanent, and can range from mild to severe. Treatment for paralysis depends on the underlying cause and can include physical therapy, medication, surgery, or other interventions.
Peripheral nervous system diseases refer to disorders that affect the peripheral nerves, which are the nerves that carry signals from the brain and spinal cord to the rest of the body. These diseases can affect the nerves themselves or the tissues surrounding them, and can result in a range of symptoms, including pain, numbness, weakness, and tingling. Some examples of peripheral nervous system diseases include: 1. Charcot-Marie-Tooth disease: A group of inherited disorders that affect the nerves in the hands and feet, causing weakness, numbness, and loss of sensation. 2. Guillain-Barre syndrome: A rare autoimmune disorder in which the body's immune system attacks the peripheral nerves, causing weakness and paralysis. 3. Peripheral neuropathy: A general term for any disorder that affects the peripheral nerves, which can be caused by a variety of factors, including diabetes, alcoholism, and exposure to certain toxins. 4. Multiple sclerosis: An autoimmune disorder that affects the central nervous system, including the brain and spinal cord, but can also affect the peripheral nerves, causing symptoms such as numbness and weakness. 5. Amyotrophic lateral sclerosis (ALS): A progressive neurodegenerative disorder that affects the nerves controlling muscle movement, leading to weakness and paralysis. Treatment for peripheral nervous system diseases depends on the specific disorder and its underlying cause. In some cases, medications or physical therapy may be used to manage symptoms, while in other cases, surgery or other interventions may be necessary.
A neuroma is a benign growth of nerve tissue that can occur in various parts of the body. It is commonly referred to as a "pinched nerve" or "nerve entrapment." Neuromas can develop when a nerve is compressed or damaged, leading to inflammation and swelling of the nerve tissue. This can cause pain, numbness, tingling, or weakness in the affected area. Neuromas can occur in different parts of the body, including the hands, feet, arms, legs, and back. They are most commonly found in the feet, particularly in the toes, where they are known as Morton's neuroma. Treatment for neuromas depends on the severity and location of the condition. In some cases, conservative treatments such as rest, ice, physical therapy, and pain medication may be sufficient. In more severe cases, surgery may be necessary to remove the neuroma or relieve pressure on the affected nerve.
Gait disorders, neurologic refer to a group of conditions that affect the way a person walks due to a neurological disorder. These disorders can be caused by a variety of factors, including damage to the nervous system, muscle weakness or spasticity, and problems with balance or coordination. Some common examples of neurologic gait disorders include Parkinson's disease, multiple sclerosis, spinal cord injuries, and stroke. These disorders can cause a range of symptoms, such as shuffling gait, difficulty with balance, tripping or falling, and changes in stride length or cadence. Treatment for neurologic gait disorders typically involves a combination of physical therapy, medication, and assistive devices, such as canes or walkers. In some cases, surgery may be necessary to address underlying neurological issues or to improve mobility.
Peripheral nerve injuries refer to damage or trauma to the nerves that are located outside of the brain and spinal cord. These nerves are responsible for transmitting signals between the central nervous system and the rest of the body, allowing us to feel sensations, move our muscles, and control our organs. Peripheral nerve injuries can occur as a result of a variety of factors, including trauma, compression, infection, or disease. Symptoms of peripheral nerve injuries can vary depending on the location and severity of the injury, but may include numbness, tingling, weakness, or loss of sensation in the affected area. Treatment for peripheral nerve injuries depends on the cause and severity of the injury. In some cases, conservative treatments such as physical therapy or medication may be sufficient to manage symptoms and promote healing. In more severe cases, surgery may be necessary to repair or replace damaged nerve tissue.
The ankle joint is a complex joint located at the lower end of the leg and the upper end of the foot. It is formed by the articulation between the talus bone of the foot and the tibia and fibula bones of the leg. The ankle joint is responsible for allowing movement in the foot and ankle, including dorsiflexion (lifting the front of the foot), plantarflexion (dropping the heel), inversion (turning the foot inward), and eversion (turning the foot outward). The ankle joint is also supported by ligaments, which help to stabilize the joint and prevent excessive movement. Injuries to the ankle joint, such as sprains or fractures, can result in pain, swelling, and limited mobility.
In the medical field, the ankle is a complex joint that connects the lower leg (tibia and fibula) to the foot (tarsus). It is made up of three bones: the talus, which sits on top of the tibia and fibula, and the calcaneus and navicular bones, which make up the heel and the base of the foot. The ankle joint is surrounded by ligaments, which provide stability and support, and by muscles and tendons, which allow movement and provide power to the foot and leg. The ankle is an important joint that allows for a wide range of motion, including dorsiflexion (lifting the front of the foot), plantarflexion (dropping the heel), inversion (turning the foot inward), and eversion (turning the foot outward). Injuries to the ankle, such as sprains and fractures, can cause pain, swelling, and limited mobility, and may require medical treatment.
In the medical field, an axon is a long, slender projection of a nerve cell (neuron) that conducts electrical impulses away from the cell body towards other neurons, muscles, or glands. The axon is covered by a myelin sheath, which is a fatty substance that insulates the axon and helps to speed up the transmission of electrical signals. Axons are responsible for transmitting information throughout the nervous system, allowing the brain and spinal cord to communicate with other parts of the body. They are essential for many bodily functions, including movement, sensation, and cognition. Damage to axons can result in a variety of neurological disorders, such as multiple sclerosis, Guillain-Barré syndrome, and peripheral neuropathy. Treatments for these conditions often focus on preserving and regenerating axons to restore normal function.
Diabetic neuropathy is a type of nerve damage that can occur as a complication of diabetes. It is caused by damage to the nerves that control movement, sensation, and other functions in the body. There are several types of diabetic neuropathy, including: 1. Peripheral neuropathy: This is the most common type of diabetic neuropathy and affects the nerves in the extremities, such as the hands, feet, and legs. It can cause numbness, tingling, pain, and weakness in the affected areas. 2. Autonomic neuropathy: This type of neuropathy affects the nerves that control automatic bodily functions, such as heart rate, digestion, and blood pressure. It can cause symptoms such as dizziness, fainting, and gastrointestinal problems. 3. Proximal neuropathy: This type of neuropathy affects the nerves in the arms and legs, causing weakness and muscle wasting in the affected areas. 4. Mononeuropathy: This is a type of neuropathy that affects a single nerve, causing symptoms such as pain, numbness, and weakness in the affected area. Diabetic neuropathy can be a serious complication of diabetes and can lead to a range of problems, including foot ulcers, infections, and even amputations. It is important for people with diabetes to manage their blood sugar levels and to see their healthcare provider regularly for monitoring and treatment.
Nerve Growth Factor (NGF) is a protein that plays a crucial role in the development and maintenance of the nervous system. It is produced by various cells, including neurons, glial cells, and some immune cells. NGF is involved in the survival, growth, and differentiation of neurons, particularly sensory neurons in the peripheral nervous system. It also plays a role in the development of the sympathetic nervous system and the enteric nervous system. In addition to its role in the nervous system, NGF has been shown to have anti-inflammatory and neuroprotective effects, and it has been studied for its potential therapeutic applications in various neurological disorders, including Alzheimer's disease, Parkinson's disease, and multiple sclerosis. NGF is also involved in the development and progression of cancer, and it has been shown to promote the growth and survival of some cancer cells. As a result, it has been targeted as a potential therapeutic target in cancer treatment.
Nerve growth factors (NGFs) are a group of proteins that play a crucial role in the development, maintenance, and repair of the nervous system. They are primarily produced by neurons and Schwann cells, which are glial cells that wrap around and support neurons. NGFs are involved in a variety of processes related to the nervous system, including the growth and survival of neurons, the regulation of synaptic plasticity, and the modulation of pain perception. They also play a role in the development of the peripheral nervous system, including the formation of sensory and motor neurons. In the medical field, NGFs have been studied for their potential therapeutic applications in a variety of neurological disorders, including Alzheimer's disease, Parkinson's disease, and traumatic brain injury. They have also been investigated as a potential treatment for peripheral neuropathy, a condition characterized by damage to the nerves that carry sensory and motor signals to and from the body's extremities.
Cranial nerves are a group of twelve pairs of nerves that emerge from the brainstem and are responsible for controlling various functions of the head and neck. These nerves are responsible for transmitting sensory information, such as touch, taste, and smell, as well as controlling movement and regulating vital functions such as heart rate and blood pressure. The cranial nerves are numbered and named according to their location and function. Some of the most well-known cranial nerves include the optic nerve (which carries visual information), the olfactory nerve (which carries information about smell), and the trigeminal nerve (which controls sensation in the face and head).
Action potentials are electrical signals that are generated by neurons in the nervous system. They are responsible for transmitting information throughout the body and are the basis of all neural communication. When a neuron is at rest, it has a negative electrical charge inside the cell and a positive charge outside the cell. When a stimulus is received by the neuron, it causes the membrane around the cell to become more permeable to sodium ions. This allows sodium ions to flow into the cell, causing the membrane potential to become more positive. This change in membrane potential is called depolarization. Once the membrane potential reaches a certain threshold, an action potential is generated. This is a rapid and brief change in the membrane potential that travels down the length of the neuron. The action potential is characterized by a rapid rise in membrane potential, followed by a rapid fall, and then a return to the resting membrane potential. Action potentials are essential for the proper functioning of the nervous system. They allow neurons to communicate with each other and transmit information throughout the body. They are also involved in a variety of important physiological processes, including muscle contraction, hormone release, and sensory perception.
Afferent pathways refer to the neural pathways that carry sensory information from the body's sensory receptors to the central nervous system (CNS), which includes the brain and spinal cord. These pathways are responsible for transmitting information about the external environment and internal bodily sensations to the CNS for processing and interpretation. Afferent pathways can be further divided into two types: sensory afferent pathways and motor afferent pathways. Sensory afferent pathways carry information about sensory stimuli, such as touch, temperature, pain, and pressure, from the body's sensory receptors to the CNS. Motor afferent pathways, on the other hand, carry information about the state of the body's muscles and organs to the CNS. Afferent pathways are essential for our ability to perceive and respond to the world around us. Any damage or dysfunction to these pathways can result in sensory deficits or other neurological disorders.
Paresis is a medical term that refers to partial paralysis, which means that a person has lost some, but not all, of the function of a muscle or group of muscles. Paresis can be caused by a variety of factors, including injury, disease, or nerve damage. It is often characterized by weakness, stiffness, or difficulty moving the affected muscles. Paresis can affect any part of the body, but it is most commonly seen in the arms and legs. It is important to note that paresis is different from paralysis, which refers to the complete loss of muscle function.
In the medical field, "cats" typically refers to Felis catus, which is the scientific name for the domestic cat. Cats are commonly kept as pets and are known for their agility, playful behavior, and affectionate nature. In veterinary medicine, cats are commonly treated for a variety of health conditions, including respiratory infections, urinary tract infections, gastrointestinal issues, and dental problems. Cats can also be used in medical research to study various diseases and conditions, such as cancer, heart disease, and neurological disorders. In some cases, the term "cats" may also refer to a group of animals used in medical research or testing. For example, cats may be used to study the effects of certain drugs or treatments on the immune system or to test new vaccines.
Blood pressure is the force exerted by the blood against the walls of the blood vessels as the heart pumps blood through the body. It is measured in millimeters of mercury (mmHg) and is typically expressed as two numbers: systolic pressure (the pressure when the heart beats) and diastolic pressure (the pressure when the heart is at rest between beats). Normal blood pressure is considered to be below 120/80 mmHg, while high blood pressure (hypertension) is defined as a systolic pressure of 140 mmHg or higher and/or a diastolic pressure of 90 mmHg or higher. High blood pressure is a major risk factor for heart disease, stroke, and other health problems.
Peroneal nerve paralysis
Sural communicating branch of common peroneal nerve
Gerdy's tubercle
Squatting position
Nerve compression syndrome
Restless legs syndrome
Broström procedure
Bagram torture and prisoner abuse
Actin
Palsy
Fibularis brevis
Functional electrical stimulation
Knee dislocation
Dorsalis pedis artery
Tendon transfer
Superior extensor retinaculum of foot
Maisonneuve fracture
Roundhouse kick
Cutaneous reflex in human locomotion
Tourniquet
Plantar reflex
Neurofibroma
Anterior tibial artery
Ankle
Extensor hallucis brevis muscle
Tumefactive multiple sclerosis
Peroneal strike
Tibialis anterior muscle
Sciatic nerve block
Extensor digitorum longus muscle
Peroneal nerve paralysis - Wikipedia
Common peroneal nerve dysfunction: MedlinePlus Medical Encyclopedia
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Ulnar3
- PNS effects were evaluated in the ulnar and peroneal nerves using measurements of maximum motor nerve conduction velocity and distal latency. (cdc.gov)
- decreased strength in muscles innervated by both ulnar nerves, both median nerves (left more than right), and both common peroneal nerves. (uab.edu)
- Subjects were examined for neuropathy, and nerve conduction was measured at the peroneal motor nerve, sural sensory nerve, and ulnar sensory and motor nerves. (cdc.gov)
Palsy11
- The origin of peroneal nerve palsy has been reported to be associated with musculoskeletal injury or isolated nerve traction and compression. (wikipedia.org)
- Most studies reported that about 30% of peroneal nerve palsy is followed from knee dislocations. (wikipedia.org)
- Treatment options for nerve palsy include both operative and non-operative techniques. (wikipedia.org)
- Causing factors of peroneal nerve palsy are such as musculoskeletal or peroneal nerve injuries. (wikipedia.org)
- For complete nerve palsy, less than 40% of patients will have complete recovery. (wikipedia.org)
- Examinations are required for following reasons: Considering lumbar radiculopathy during the examination Possibility of foot drop Sensory loss that may be difficult to determine because of variable and small autonomous zone of sensation Tinel's sign over the fibular neck that can help localize the site of nerve compression Checking for direct compression that reproduces nerve symptoms Electromyography is used to observe peroneal nerve palsy within one month of injuries. (wikipedia.org)
- And if it is partial peroneal nerve palsy, patients have higher chance to recover fully from the palsy. (wikipedia.org)
- In this case, despite complete palsy of the common peroneal nerve following the injury, the patient had near full motor recovery. (medscape.com)
- It was reported the patient experienced severe pain and disability secondary to post operative radial nerve palsy. (fda.gov)
- Peroneal nerve palsy is usually caused by compression of the nerve against the lateral aspect of the fibular neck. (msdmanuals.com)
- Peroneal nerve palsy causes footdrop (weakened dorsiflexion and eversion of the foot) and, occasionally, a sensory deficit in the anterolateral aspect of the lower leg and the dorsum of the foot or in the web space between the 1st and 2nd metatarsals. (msdmanuals.com)
Lesions4
- This is not surprising given the strong evidence for interaction between myelin and axon gene expression in development and after experimental nerve lesions. (medscape.com)
- Two patients developed temporary superficial peroneal nerve lesions. (aott.org.tr)
- There was no objective sensory finding and no cranial nerve lesions. (who.int)
- There was no objective sensory increased to 23 kg/m², he recovered finding and no cranial nerve lesions. (who.int)
Ankle13
- Peroneal nerve paralysis is a paralysis on common fibular nerve that affects patient's ability to lift the foot at the ankle. (wikipedia.org)
- Peroneal nerve is most commonly interrupted at the knee and possibly at the joint of hip and ankle. (wikipedia.org)
- At the level of ankle, the superficial peroneal nerve splits to fan out between the medial and lateral malleoli. (medscape.com)
- As part of an ankle block required to manipulate a fracture or dislocated ankle (A combination of posterior tibial , saphenous , superficial peroneal, deep peroneal, and sural nerve blocks results in complete block of sensory perception beneath the ankle. (medscape.com)
- This block requires anesthetization of 5 nerves for complete sensory block below the ankle. (medscape.com)
- The areas to anesthetize include a line along the anterior ankle for the superficial peroneal nerve (blue line), the deep peroneal nerve (red star), the saphenous nerve (pink star), the sural nerve (green arrow), and the posterior tibial nerve (orange arrow). (medscape.com)
- Deep peroneal nerve block is one of the 2 deep nerve blocks at the level of the ankle. (medscape.com)
- The deep peroneal nerve block is easy to perform and may constitute part of an ankle block. (medscape.com)
- [ 1 , 2 ] In the distal one third of the ankle, the nerve is located between the tibialis anterior and extensor hallucis longus muscles and is superficial to the anterior tibial artery. (medscape.com)
- At an average distance of 12.5 mm proximal to the ankle, the nerve crosses deep to the extensor hallucis longus tendon and courses between the extensor hallucis longus and extensor digitorum longus tendons. (medscape.com)
- Treatment of acute or chronic pain conditions involving the foot with entrapment of the deep peroneal nerve at the anterior tarsal tunnel (Anterior tarsal tunnel syndrome is characterized by persistent aching of the dorsum of the foot that is worse at night and is made better by moving the affected toes and ankle. (medscape.com)
- Often, peroneal nerve injuries develop because of a traumatic injury to your knee, leg or ankle. (clevelandclinic.org)
- O'Malley can not dorsiflex his right foot due to the momentary nerve injury which then causes him to roll his right ankle later. (sportskeeda.com)
Superficial peron6
- Regional block of the superficial peroneal nerve allows for rapid anesthetization of the dorsum of the foot, which allows for management of lacerations, fractures, nail bed injuries, or other pathology involving the dorsum of the foot. (medscape.com)
- Understanding the anatomical distribution of the superficial peroneal nerve is helpful in performing a successful blockade of this nerve. (medscape.com)
- The superficial peroneal nerve arises from the common peroneal nerve, which also gives rise to the deep peroneal nerve. (medscape.com)
- The superficial peroneal nerve originates between the peroneus longus muscle and the fibula. (medscape.com)
- Dermatome of the superficial peroneal nerve at the level of posterior calf. (medscape.com)
- Superficial peroneal nerve dermatome at the level of the anterior lower leg. (medscape.com)
Sciatic nerve7
- The peroneal nerve is a branch of the sciatic nerve. (medlineplus.gov)
- It's a branch of the sciatic nerve that travels from the back of your thigh along the outside of your knee to the front of your lower leg. (clevelandclinic.org)
- Injuries to your sciatic nerve in your buttock or back of your thigh can also cause a foot drop. (clevelandclinic.org)
- Sciatic nerve transection, early after birth, results in significant degeneration of spinal motoneurons as well as sensory neurons present in the dorsal root ganglia. (hindawi.com)
- The main objective of the study was to determine the level of bifurcation of the sciatic nerve above the transverse popliteal crease among Ugandans at Mulago Hospital Complex. (bvsalud.org)
- Conclusions: The Bifurcation of the sciatic nerve occurs at variable distances from the transverse popliteal crease and appreciation of these variations is essential. (bvsalud.org)
- More than 22of all nerves leave the pelvis as two separate nerves and therefore the sciatic nerve trunk cannot be wholly traced or used for anesthetic block in the gluteal region or thigh for procedures in the leg and foot. (bvsalud.org)
Dysfunction6
- Dysfunction of a single nerve such as the common peroneal nerve is called a mononeuropathy. (wikipedia.org)
- Common peroneal nerve dysfunction is due to damage to the peroneal nerve leading to loss of movement or sensation in the foot and leg. (medlineplus.gov)
- This condition is also called common fibular nerve dysfunction. (medlineplus.gov)
- Common peroneal nerve dysfunction is a type of peripheral neuropathy (nerve damage outside the brain or spinal cord). (medlineplus.gov)
- Other tests may be done depending on the suspected cause of nerve dysfunction, and the person's symptoms and how they develop. (medlineplus.gov)
- Successfully treating the cause may relieve the dysfunction, but it may take several months for the nerve to improve. (medlineplus.gov)
Muscle action potentials1
- There was a significant reduction in the ratio of amplitudes of muscle action potentials measured during peroneal nerve stimulation. (cdc.gov)
Electromyography2
- Nerve conduction studies and electromyography are very useful in finding where the nerve problem is. (clevelandclinic.org)
- Electromyography demonstrated impairment of bilateral peroneal nerve function: evoked amplitude was markedly reduced, with slight slowing of nerve conduction velocity. (who.int)
Fibula3
- It runs anterolateral to the fibula between the peroneal muscles and the extensor digitorum longus, eventually supplying the peroneal muscles. (medscape.com)
- The common peroneal nerve (root values: L4, L5, S1, and S2) winds around the fibula through the fibular tunnel. (medscape.com)
- The deep peroneal nerve (or the anterior tibial nerve) begins at the bifurcation of the common peroneal nerve, between the fibula and upper part of peroneus longus. (medscape.com)
Dorsal digita2
- The nerve splits into the medial dorsal cutaneous nerve and the intermediate dorsal cutaneous nerve, which give rise to the dorsal digital nerves. (medscape.com)
- At the first interosseous space, it divides into dorsal digital nerves, which provide sensory innervation to the first webspace and the adjacent dorsum of the foot. (medscape.com)
Myelin sheath2
- Damages on peroneal nerves destroy the myelin sheath that covers the axon or the whole nerve cell. (wikipedia.org)
- Damage to the nerve disrupts the myelin sheath that covers the axon (branch of the nerve cell). (medlineplus.gov)
Muscles4
- There might be a loss of feeling, muscle control, muscle tone, and eventual loss of muscle mass because the nerves aren't stimulating the muscles after they are damaged. (wikipedia.org)
- Foot drop has several possible causes, typically due to an issue with your nerves and/or muscles. (clevelandclinic.org)
- and from the intermuscular septa between it and the tibialis anterior on the medial, and the peroneal muscles on the lateral side. (wikipedia.org)
- It results from compression of the median nerve in the volar aspect of the wrist between the transverse superficial carpal ligament and the flexor tendons of the forearm muscles. (msdmanuals.com)
Injuries3
- However, certain conditions may also cause single nerve injuries. (wikipedia.org)
- Certain body-wide conditions can also cause single nerve injuries. (medlineplus.gov)
- Multi-ligament knee injury is a complex and difficult injury to manage, particularly when there are associated nerve or vascular injuries. (medscape.com)
Neurolysis3
- As a prelude to neurolysis or radiofrequency ablation of the deep peroneal nerve. (medscape.com)
- During the same procedure, another surgeon (AD) performed neurolysis of the common peroneal nerve. (medscape.com)
- The peroneal nerve was extremely scarred to the surrounding tissue and extensive, meticulous common peroneal neurolysis was performed. (medscape.com)
Brain and spinal cord1
- Your peroneal nerve is a peripheral nerve (nerves outside your brain and spinal cord). (clevelandclinic.org)
Extensor1
- In the leg, the deep peroneal nerve supplies muscular branches to the tibialis anterior, extensor digitorum longus, peroneus tertius, and extensor hallucis longus. (medscape.com)
Tibial nerve8
- Tibial nerve motor function and sensation were normal. (medscape.com)
- Furthermore, evoked spinal reflex responses of the soleus muscle (H-reflex evoked at rest and during iMVC, V-wave), peak twitch torques induced by electrical stimulation of the posterior tibial nerve at rest and fatigue resistance were evaluated. (frontiersin.org)
- These evoked potentials are elicited by electrical stimulation of the posterior tibial nerve in the popliteal fossa and their amplitudes can be recorded in the soleus muscle (SOL). (frontiersin.org)
- The tibial nerve and common peroneal nerve (also known as common fibular nerve) originate at L5, S1 and S2. (physio-pedia.com)
- The tibial nerve provides motor fibres to gastrocnemius, soleus, tibialis posterior, flexor digitorum longus, and flexor hallucis longus. (physio-pedia.com)
- Differences between brain responses to peroneal electrical transcutaneous neuromodulation and transcutaneous tibial nerve stimulation, two treatments for overactive bladder. (urotoday.com)
- Real-time changes in brain activity during tibial nerve stimulation for overactive bladder: Evidence from functional near-infrared spectroscopy hype scanning. (urotoday.com)
- Transcutaneous tibial nerve electrical stimulation versus vaginal electrical stimulation in women with overactive bladder syndrome: Is there a role for short-term interventions? (urotoday.com)
Peripheral neuropathy1
- Any condition that affects peripheral nerves, such as Charcot-Marie-Tooth disease or acquired peripheral neuropathy , can affect your peroneal nerve and cause foot drop. (clevelandclinic.org)
Peroneus1
- The superficial branch of the common peroneal nerve sends motor fibres to peroneus (fibularis) longus and brevis. (physio-pedia.com)
Lateral2
- However, this nerve does not supply the web space between the first and second digits or the lateral fifth digit. (medscape.com)
- Toenail repair (Toenail repair on the lateral first digit and medial second digit also requires deep peroneal nerve block. (medscape.com)
Common8
- Relatively tethered location around fibular head, tenuous vascular supply and epineural connective tissues are possible factors that cause damage on the common peroneal nerve. (wikipedia.org)
- Common peroneal nerve is a type of mononeuropathy . (medlineplus.gov)
- Motor function in the distribution of the common peroneal nerve was absent. (medscape.com)
- The most common causes are peroneal nerve injury and lumbar radiculopathy. (clevelandclinic.org)
- Peroneal nerve injury is a common cause of the symptom. (clevelandclinic.org)
- Your common peroneal nerve is a peripheral nerve that extends down your leg. (clevelandclinic.org)
- Several things can damage your common peroneal nerve and cause foot drop. (clevelandclinic.org)
- Moreover, INIs are also a common source of medicolegal litigation with 60% of INI complications during thyroid surgery leading to malpractice lawsuits and 82% of cases of spinal accessory nerve injury resulting in patient compensation 17 , 18 . (nature.com)
Anesthesia1
- The deep peroneal nerve block is useful for anesthesia and postoperative analgesia to surgeries of the first web space (eg, Morton neuroma ). (medscape.com)
Tibialis1
- Between it and the tibialis anterior are the upper portions of the anterior tibial vessels and deep peroneal nerve. (wikipedia.org)
Sensory and motor1
- The diagnosis of GBS is clinical but may be aided by electrophysiology which is also important to characterise the two main electrophysiological subtypes: acute inflammatory demyelinating polyradiculoneuropathy (AIDP), which is sensory and motor and displays demyelinating changes on nerve conduction studies, and acute motor axonal neuropathy (AMAN), which is primarily axonal and thought to be purely motor. (bmj.com)
Distal3
- These nerves supply the skin of the anterolateral distal third of the leg, most of the dorsal foot, and the digits. (medscape.com)
- Just distal to the fibular tunnel, the nerve divides into the superficial and deep peroneal nerves. (medscape.com)
- Distal latency, residual latency, and muscle or nerve action potential amplitudes did not differ significantly in the study groups. (cdc.gov)
Eversion1
- One year after surgery, he had normal, symmetrical peroneal eversion strength with slight weakness of dorsiflexion power on the left. (medscape.com)
Pelvis1
- The nerves bifurcated in the gluteal region and posterior thigh in 62 cadavers (77.5) and 18 in the pelvis (22.5). (bvsalud.org)
Stimulation2
- In surgical procedures where the risk of accidental nerve damage is prevalent, surgeons commonly use electrical stimulation (ES) during intraoperative nerve monitoring (IONM) to assess a nerve's functional integrity. (nature.com)
- IONM seeks to preserve peripheral nerve function through electrical stimulation (ES) of at risk nerves throughout surgery and examining any changes in the amplitude and latency of the evoked signals that are indicative of damage. (nature.com)
Cranial1
- It includes the cranial nerves and spinal nerves from their origin to their end. (msdmanuals.com)
Neuromuscular2
- Peroneal nerve paralysis usually leads to neuromuscular disorder, peroneal nerve injury, or foot drop which can be symptoms of more serious disorders such as nerve compression. (wikipedia.org)
- Over 3500 patients are studied in the EMG laboratory each year with a full variety of studies ranging from simple nerve conduction studies to complex studies of neuromuscular transmission or the central motor pathways. (lhsc.on.ca)
Knee2
- Peroneal nerve injury occurs when the knee is exposed to various stress. (wikipedia.org)
- Looks like a hit to the peroneal nerve around the right knee. (sportskeeda.com)
Entrapment1
- Compression of nerves in narrow passageways causes entrapment neuropathy (eg, in carpal tunnel syndrome). (msdmanuals.com)
Anatomy1
- hence the need for healthcare workers to have adequate appreciation of the applied anatomy of the nerve. (bvsalud.org)
Cervical1
- This is the area innervated by the 6th cervical nerve from your neck. (healthtap.com)
Arises1
- Peroneal nerve in continuity arises from defined cause will be recovered better than those arise from unknown causes. (wikipedia.org)
Dorsiflexion1
- At 6 months, he began to have early return of peroneal motor function, specifically dorsiflexion. (medscape.com)
Sural nerve1
- The sensory conduction velocity was measured in the sural nerve. (cdc.gov)
Conduction velocities4
- In the late 1960s, neurophysiologic testing allowed the classification of CMT into 2 groups, one with slow nerve conduction velocities and histologic features of a hypertrophic demyelinating neuropathy (hereditary motor and sensory neuropathy type 1 or CMT1) and another with relatively normal velocities and axonal and neuronal degeneration (hereditary motor and sensory neuropathy type 2 or CMT2). (medscape.com)
- The effects of arsenic (7440382) on nerve conduction velocities were studied in Alaskan residents with naturally contaminated drinking water. (cdc.gov)
- One or more nerve conduction velocities were abnormal in 13 subjects, but nerve velocity measurements were not related to estimated daily arsenic ingestion or arsenic concentrations in water or urine. (cdc.gov)
- The authors conclude that nerve conduction velocities are insensitive in screening for subclinical neuropathy in subjects exposed to inorganic arsenic. (cdc.gov)
Mononeuropathy2
- Mononeuropathy means the nerve damage is occurred in one area. (wikipedia.org)
- Mononeuropathy is nerve damage to a single nerve. (medlineplus.gov)
Deep5
- Deep peroneal nerve and adjacent structures. (medscape.com)
- The deep peroneal branch continues into the foot along with the tibial artery and the vein. (medscape.com)
- Sensation in the distribution of the deep and superficial peroneal nerves was decreased, but the patient could detect light touch in those distributions. (medscape.com)
- Deep peroneal nerve (L4, L5, S1). (getbodysmart.com)
- occasionally supply the area typically innervated by the deep peroneal nerve. (physio-pedia.com)
Bifurcation1
- the nerves reunited before the final bifurcation occurred in the thigh. (bvsalud.org)
Injury1
- Experimentally, a well-accepted model to mimic axotomy injury retrograde repercussion to spinal neurons is the neonatal peripheral nerve axotomy [ 13 - 18 ]. (hindawi.com)
Affects1
- Hansen's disease is an infectious, chronic bacterial disease that primarily affects the skin and peripheral nerves. (cdc.gov)
Weakness2
- Single mononeuropathies are characterized by sensory disturbances and weakness in the distribution of the affected peripheral nerve. (msdmanuals.com)
- pure sensory nerve involvement begins with sensory disturbances and no weakness. (msdmanuals.com)
Symptoms1
- The authors conclude that symptoms of peripheral nerve polyneuritis are not manifested at CS2 exposure concentrations below 10ppm. (cdc.gov)
Compression2
- If the symptom does not get any better in few months, surgery is required to decompress the nerve compression. (wikipedia.org)
- Compression of a nerve may be transient (eg, caused by an activity) or fixed (eg, caused by a mass or anatomic abnormality). (msdmanuals.com)
Tumor2
- Sometimes, a tumor or cyst in your peroneal nerve can cause a foot drop. (clevelandclinic.org)
- Hemorrhage that compresses a nerve, exposure to cold or radiation, or direct tumor invasion may also cause neuropathy. (msdmanuals.com)
Motor3
- Overall, the present data suggest that acute repair of neonatal peripheral nerves with fibrin sealant results in neuroprotection and regeneration of motor and sensory axons. (hindawi.com)
- The results of median sensory-motor, sural sensory and post-tibial motor nerve conduction studies were normal. (who.int)
- The results antituberculosis pharmacotherapy is had no history of immunodeficiency, of median sensory-motor, sural sen- relatively uncommon, although the fre- no diabetes, no renal failure, no hepatic sory and post-tibial motor nerve con- quency of the usage of antituberculosis failure, no HIV infection and he was a duction studies were normal. (who.int)
Median nerve1
- Transient forearm conduction block in the median nerve. (lhsc.on.ca)
Branches1
- From the enlargement, 3 minute interosseous branches (dorsal interosseous nerves) are given off, which supply the tarsal joints and the metatarsophalangeal joints of the 2nd, 3rd, and 4th toes. (medscape.com)
Neuropathy1
- Only one subject, a diabetic, had both abnormal nerve velocities and signs of neuropathy. (cdc.gov)
Severe1
- Severe nerve damage may cause permanent disability. (medlineplus.gov)