Plant Poisoning
Swainsonine
Aversive Therapy
Astragalus Plant
Five triterpene glycosides from Oxytropis myriophylla. (1/16)
Four new triterpene glycosides: one cycloartane-type glycoside and three azukisapogenol glycosides were isolated together with one known oleanene bisdesmoside from the Mongolian natural medicine, Oxytropis myriophylla. (+info)Appearance and disappearance of swainsonine in serum and milk of lactating ruminants with nursing young following a single dose exposure to swainsonine (locoweed; Oxytropis sericea). (2/16)
A series of experiments were conducted to investigate the elimination of swainsonine in the milk of lactating ruminants following a single dose oral exposure to swainsonine (locoweed; Oxytropis sericea) and to assess subsequent subclinical effects on the mothers and their nursing young. In a preliminary experiment, lactating ewes were gavaged with locoweed providing 0.8 mg swainsonine/kg BW (n = 4; BW = 75.8 +/- 3.6 kg; lactation = d 45) and lactating cows were offered up to 2.0 mg swainsonine/kg BW free choice (n = 16; BW = 389.6 +/- 20.9 kg; lactation = d 90). Serum and milk were collected at h 0 (before treatment), 3, 6, 12, and 24 for ewes, and h 0 (before treatment), 6, 12, 18, and 24 for cows. Swainsonine was highest (P < 0.05) by h 6 in the serum and milk of ewes. Consumption of at least 0.61 mg swainsonine/kg BW induced consistent (> 0.025 microg/mL) appearance of swainsonine in cow serum and milk. In response to the results obtained in the preliminary experiment, a subsequent experiment utilizing lactating ewes (n = 13; BW = 74.8 +/- 6.4 kg; lactation = d 30) and cows (n = 13; BW = 460.8 +/- 51.9 kg; lactation = d 90) was conducted. Each lactating ruminant was gavaged with a locoweed extract to provide 0 (control), 0.2, or 0.8 mg swainsonine/kg BW and individually penned with her nursing young. Serum and milk from the mothers and serum from the nursing young were collected at h 0 (before treatment), 3, 6, 9, 12, 24 and 48 (an additional sample was obtained at h 72 for ewes and lambs). Serum and milk swainsonine was higher (P < 0.05) in the 0.8 mg treated groups and maximal (P < 0.05) concentrations occurred from h 3 to 6 for ewes and h 6 to 12 h for cows (P < 0.05). Rises in alkaline phosphatase activity indicated subclinical toxicity in the treated ewes (P < 0.05). Following a single dose oral exposure to 0.2 and 0.8 mg swainsonine/kg BW provided by a locoweed extract, swainsonine was detected in the serum and milk of lactating ewes and cows, and rises in serum alkaline phosphatase activity were observed in the ewes. Neither swainsonine nor changes in alkaline phosphatase activity was detected in the serum of the lambs and calves nursing the ewes and cows dosed with swainsonine. (+info)Dose response of sheep poisoned with locoweed (Oxytropis sericea). (3/16)
Locoweed poisoning occurs when livestock consume swainsonine-containing Astragalus and Oxytropis species over several weeks. Although the clinical and histologic changes of poisoning have been described, the dose or duration of swainsonine ingestion that results in significant or irreversible damage is not known. The purpose of this research was to document the swainsonine doses that produce clinical intoxication and histologic lesions. Twenty-one mixed-breed wethers were dosed by gavage with ground Oxytropis sericea to obtain swainsonine doses of 0.0, 0.05, 0.1, 0.2, 0.4, 0.8, and 1.0 mg/kg/day for 30 days. Sheep receiving > or = 0.2 mg/kg gained less weight than controls. After 16 days, animals receiving > or = 0.4 mg/kg were depressed, reluctant to move, and did not eat their feed rations. All treatment groups had serum biochemical changes, including depressed alpha-mannosidase, increased aspartate aminotransferase and alkaline phosphatase, as well as sporadic changes in lactate dehydrogenase, sodium, chloride, magnesium, albumin, and osmolarity. Typical locoweed-induced cellular vacuolation was seen in the following tissues and swainsonine doses: exocrine pancreas at > or = 0.05 mg/kg; proximal convoluted renal and thyroid follicular epithelium at > or = 0.1 mg/kg; Purkinje's cells, Kupffer's cells, splenic and lymph node macrophages, and transitional epithelium of the urinary bladder at > or = 0.2 mg/kg; neurons of the basal ganglia, mesencephalon, and metencephalon at > or = 0.4 mg/kg; and cerebellar neurons and glia at > or = 0.8 mg/kg. Histologic lesions were generally found when tissue swainsonine concentrations were approximately 150 ng/g. Both the clinical and histologic lesions, especially cerebellar lesions are suggestive of neurologic dysfunction even at low daily swainsonine doses of 0.2 mg/kg, suggesting that prolonged locoweed exposure, even at low doses, results in significant production losses as well as histologic and functional damage. (+info)New flavonoids from Oxytropis myriophylla. (4/16)
Eight compounds were isolated from Oxytropis myriophylla. On the basis of spectral analyses, their structures were elucidated to be (6R,9R)-roseoside (1), (6R,9S)-roseoside (2), adenosine (3), myriophylloside B (4), myriophylloside C (5), myriophylloside D (6), myriophylloside E (7), and myriophylloside F (8). Five flavonoids (4-8) were new compounds, and the three known compounds were isolated from this plant for the first time. (+info)The toxicosis of Embellisia fungi from locoweed (Oxytropis lambertii) is similar to locoweed toxicosis in rats. (5/16)
Locoweeds cause significant livestock poisoning and economic loss in the western United States. The toxicity of Embellisia sp. fungi isolated from locoweed was compared with locoweed toxicity using the rat as a model. Rats were fed diets containing locoweed, fungus and alfalfa, or alfalfa. Locoweed- and fungus-fed rats consumed swainsonine-containing food at approximately 1.3 mg x kg(-1) x d(-1), gained less weight (P = 0.001) and ate less than controls. Swainsonine is the principal agent responsible for inducing locoism in animals. The concentrations of alkaline phosphatase and aspartate aminotransferase enzymes were greater (P < 0.05) in serum of locoweed- and fungus-fed rats compared with control rats. Similar intracellular vacuolation was observed in renal, pancreatic, and hepatic tissues of rats that consumed either locoweed or fungus. Rats that ate locoweed or Embellisia fungi displayed indistinguishable toxicity symptoms. The Embellisia fungi from locoweed can induce toxicity without the plants. Locoism management strategies need to involve management of the Embellisia fungi. (+info)Effects of locoweed on serum swainsonine and selected serum constituents in sheep during acute and subacute oral/intraruminal exposure. (6/16)
A study was conducted to evaluate the effects of acute and subacute locoweed exposure on serum swainsonine concentrations and selected serum constituents in sheep. Thirteen mixed-breed wethers (BW = 47.5 +/- 9.3 kg) were assigned randomly to 0.2, 0.4, or 0.8 mg of swainsonine x kg BW(-1) x d(-1) treatments. During acute (24 h) and subacute (19 d) exposure, serum swainsonine was detected in all treatments and was greatest (P < 0.03) in the 0.8 mg treatment. Serum alkaline phosphate (ALK-P) activity was increased (P < 0.01) for the 0.8 mg treatment compared with baseline (0 h) by 7 h and continued to increase throughout the initial 22 h following acute exposure to locoweed. A linear increase (P < 0.01) in serum ALK-P activity was noted, with the rate being 3.00 +/- 0.56 U x L(-1) x h(-1). Serum ALK-P activity was increased (P < 0.05) across treatments on d 7 over d -19, -12, 0, 1, 21, and 26; on d 14 over d -19, -12, 0, and 26; and on d 19 over d -19, -12, 0, 1, 21, and 26. By d 20, approximately 48 h after last exposure to swainsonine, serum ALK-P activities were no longer different (P = 0.13) than baseline (d -19, -12, and 0), and by d 26 values had generally returned to baseline. No linear (P = 0.98), quadratic (P = 0.63), or cubic effects of swainsonine with time from exposure were noted for serum aspartate aminotransferase. Similar to serum ALK-P activities, serum aspartate aminotransferase activities were increased (P < 0.05) across treatment levels on d 7, 14, 19, 20, 21, and 26 over those on d -19, -12, 0, and 1. Total serum Fe was decreased (P < 0.05) within the initial 22 h following the swainsonine exposure. On d 21 (48 h after swainsonine feeding ended), serum Fe increased to 472 mg/L. Concentrations of ceruloplasmin were lower (P < 0.10) on d 14 and 19 following exposure to locoweed. Recovery of ceruloplasmin levels coincided with similar changes in serum Fe. There was a linear (slope = 0.33 mg x dL(-1) x d(-1); P < 0.01) effect with time of exposure to locoweed (i.e., swainsonine) on serum triglyceride concentrations. Rapid changes in serum ALK-P and Fe concentrations without parallel changes in other damage markers indicate that acute exposure to swainsonine induces metabolic changes that may impair animal production and health before events of cytotoxicity thought to induce clinical manifestation of locoism. (+info)The effect of alkaloid from Oxytropis ochrocephala on growth inhibition and expression of PCNA and p53 in mice bearing H22 Hepatocellular Carcinoma. (7/16)
To study the antitumor activity of alkaloid extracted from Oxytropis ochrocephala and its possible mechanism, we observed the effect of alkaloid on tumor weight and expression of PCNA and p53 in mice bearing H(22) hepatocellular carcinoma by means of immunohistochemistry SP method. After treatment with alkaloid from Oxytropis ochrocephala, the results showed that alkaloid administration (25 and 50 mg/kg body weight, p.o.) could inhibit H(22) hepatocellular carcinoma growth to various extent, and the rates of inhibition were 48.5% and 57.7% respectively (p<0.01). The antitumor activity of the alkaloid is in a dose dependent manner, with no signs of toxicity to weight, kidney and liver. The sections of tumor showed the number of tumor cell decreased and nucleus appeared putrescence such as nucleus atrophy, disintegrating and dissolving. Meanwhile, the expression of PCNA and mutant p53 protein positive cell numbers in mice bearing H(22) hepatocellular carcinoma also suppressed by alkaloid (p<0.05). It suggested that Alkaloid from Oxytropis ochrocephala showed antitumor effect and its possible mechanism might be associated with the expression inhibition of PCNA and mutant p53 protein. Further studies are needed to explore the antitumor activity of the other compounds of Oxytropis ochrocephala and to specify their possible mechanism of action. (+info)Locoweed (Oxytropis sericea)-induced lesions in mule deer (Odocoileius hemionus). (8/16)
Locoweed poisoning has been reported in wildlife, but it is unknown whether mule deer (Odocoileius hemionus) are susceptible. In areas that are heavily infested with locoweed, deer and elk (Cervus elaphus nelsoni) have developed a spongiform encephalopathy, chronic wasting disease (CWD). Although these are distinct diseases, no good comparisons are available. The purpose of this study was to induce and describe chronic locoweed poisoning in deer and compare it with the lesions of CWD. Two groups of four mule deer were fed either a complete pelleted ration or a similar ration containing 15% locoweed (Oxytropis sericea). Poisoned deer lost weight and developed a scruffy, dull coat. They developed reluctance to move, and movement produced subtle intention tremors. Poisoned deer had extensive vacuolation of visceral tissues, which was most severe in the exocrine pancreas. Thyroid follicular epithelium, renal tubular epithelium, and macrophages in many tissues were mildly vacuolated. The exposed deer also had mild neuronal swelling and cytoplasmic vacuolation that was most obvious in Purkinje cells. Axonal swelling and dystrophy was found in many white tracts, but it was most severe in the cerebellar peduncles and the gracilis and cuneate fasciculi. These findings indicate that deer are susceptible to locoweed poisoning, but the lesions differ in severity and distribution from those of other species. The histologic changes of locoweed poisoning are distinct from those of CWD in deer; however, the clinical presentation of locoweed poisoning in deer is similar. Histologic and immunohistochemical studies are required for a definitive diagnosis. (+info)Oxytropis is a genus of flowering plants in the legume family, Fabaceae. It is native to temperate regions of the Northern Hemisphere, primarily in North America and Asia. Some common names for Oxytropis include locoweeds and wild peas.
In a medical context, Oxytropis species are most well-known for containing toxic alkaloids that can cause serious poisoning in livestock, particularly cattle, sheep, and goats. The toxins, including swainsonine and other indolizidine alkaloids, can affect the nervous system and cause symptoms such as weakness, tremors, blindness, and ultimately death.
While Oxytropis poisoning is not a direct concern for human health, it is important for medical professionals to be aware of its potential impact on animal health in rural and agricultural communities.
Plant poisoning is a form of poisoning that occurs when someone ingests, inhales, or comes into contact with any part of a plant that contains toxic substances. These toxins can cause a range of symptoms, depending on the type and amount of plant consumed or exposed to, as well as the individual's age, health status, and sensitivity to the toxin.
Symptoms of plant poisoning may include nausea, vomiting, diarrhea, abdominal pain, difficulty breathing, skin rashes, seizures, or in severe cases, even death. Some common plants that can cause poisoning include poison ivy, poison oak, foxglove, oleander, and hemlock, among many others.
If you suspect plant poisoning, it is important to seek medical attention immediately and bring a sample of the plant or information about its identity if possible. This will help healthcare providers diagnose and treat the poisoning more effectively.
Swainsonine is not a medical condition or disease, but rather a toxin that can cause a medical condition known as "locoism" in animals. Swainsonine is produced by certain plants, including some species of the genera Swainsona and Astragalus, which are commonly known as locoweeds.
Swainsonine inhibits an enzyme called alpha-mannosidase, leading to abnormal accumulation of mannose-rich oligosaccharides in various tissues and organs. This can result in a range of clinical signs, including neurological symptoms such as tremors, ataxia (loss of coordination), and behavioral changes; gastrointestinal symptoms such as diarrhea, weight loss, and decreased appetite; and reproductive problems.
Locoism is most commonly seen in grazing animals such as cattle, sheep, and horses that consume large quantities of locoweeds over an extended period. It can be difficult to diagnose and treat, and prevention through management practices such as rotational grazing and avoiding the introduction of toxic plants into pastures is often the best approach.
Aversive therapy is a behavioral treatment approach that uses negative reinforcement or punishment to help an individual reduce or stop undesirable behaviors. The goal of aversive therapy is to condition the person to associate the undesirable behavior with an unpleasant stimulus, such as a taste, sound, or image, so that they are deterred from engaging in the behavior in the future.
In aversive therapy, the therapist may use several techniques, including:
1. Contingent negative reinforcement: This involves removing a positive reinforcer (a reward) after the undesirable behavior occurs. For example, if a child with a disruptive behavior disorder is given tokens for good behavior that can be exchanged for prizes, and then loses tokens for misbehaving, this is an example of contingent negative reinforcement.
2. Punishment: This involves presenting an unpleasant stimulus immediately after the undesirable behavior occurs. For example, if a person who bites their nails receives a mild electric shock every time they bite their nails, this is an example of punishment.
3. Avoidance conditioning: This involves associating a negative stimulus with a particular situation or object to create an aversion to it. For example, if a person has a phobia of spiders, the therapist may gradually expose them to images or objects associated with spiders while also presenting a mild electric shock. Over time, the person learns to associate the spider-related stimuli with the unpleasant shock and develops an aversion to spiders.
It's important to note that aversive therapy can be controversial due to concerns about potential harm, including physical discomfort or psychological distress. As a result, it is typically used as a last resort when other treatment approaches have been ineffective, and only under the close supervision of a qualified professional who can ensure that the therapy is administered safely and ethically.
Astragalus membranaceus, also known as Astragalus propinquus, is a plant that is native to China and has been used in traditional Chinese medicine for centuries. It is often referred to simply as "astragalus" and its root is used in herbal remedies.
In traditional Chinese medicine, astragalus is considered to have warming and drying properties, and is often used to strengthen the body's defenses, or "wei qi," which is believed to help protect against external pathogens. It is also used to treat a variety of conditions, including fatigue, weakness, and respiratory infections.
In modern scientific research, astragalus has been studied for its potential immune-boosting, anti-inflammatory, and antioxidant effects. Some studies have suggested that it may help to improve immune function, reduce inflammation, and protect against oxidative stress. However, more research is needed to confirm these potential benefits and determine the optimal dosage and safety of astragalus supplements.
It's important to note that astragalus should not be used as a substitute for conventional medical treatment, and anyone considering taking it as a supplement should speak with their healthcare provider first to discuss the potential risks and benefits.
I'm happy to help, but I want to clarify that the term "sheep diseases" refers to a wide range of health issues that can affect sheep populations. It's not a single medical condition with a specific definition. Here are some examples of common diseases that can affect sheep:
1. Ovine Johne's Disease (OJD): A chronic, infectious disease caused by the bacterium Mycobacterium avium subspecies paratuberculosis. It affects the intestines and can cause weight loss, diarrhea, and death.
2. Footrot: A highly contagious bacterial infection that affects the feet of sheep, causing lameness, swelling, and pain. It's caused by the bacteria Dichelobacter nodosus.
3. Caseous Lymphadenitis (CL): A chronic infectious disease caused by the bacterium Corynebacterium pseudotuberculosis. It affects the lymph nodes and can cause abscesses, weight loss, and death.
4. Contagious Ecthyma (Orf): A highly contagious viral infection that affects the skin and mucous membranes of sheep, causing sores and lesions.
5. Mastitis: An inflammation of the mammary gland in sheep, usually caused by a bacterial infection. It can cause decreased milk production, fever, and loss of appetite.
6. Pneumonia: A respiratory infection that can affect sheep, causing coughing, difficulty breathing, and fever. It can be caused by various bacteria or viruses.
7. Enterotoxemia: A potentially fatal disease caused by the overproduction of toxins in the intestines of sheep, usually due to a bacterial infection with Clostridium perfringens.
8. Polioencephalomalacia (PEM): A neurological disorder that affects the brain of sheep, causing symptoms such as blindness, circling, and seizures. It's often caused by a thiamine deficiency or excessive sulfur intake.
9. Toxoplasmosis: A parasitic infection that can affect sheep, causing abortion, stillbirth, and neurological symptoms.
10. Blue tongue: A viral disease that affects sheep, causing fever, respiratory distress, and mouth ulcers. It's transmitted by insect vectors and is often associated with climate change.
Oxytropis
Oxytropis lambertii
Oxytropis sobolevskajae
Wollastonaria oxytropis
Oxytropis pilosa
Oxytropis campestris
Polystira oxytropis
Basilissopsis oxytropis
Oxytropis splendens
Zygaena oxytropis
Oxytropis urumovii
Oxytropis prenja
Oxytropis sericea
Oxytropis sordida
Oxytropis kozhuharovii
Oxytropis podocarpa
List of Oxytropis species
Vihren
Galegeae
Discula
Hystricella
List of non-marine molluscs of Madeira
List of non-marine molluscs of Myanmar
Penstemon gibbensii
Rove Formation
Agriades glandon
Igor Lintchevski
List of the vascular plants in the Red Data Book of Russia
Indigofera
List of Fabaceae genera
Oxytropis - Wikipedia
Western USA wildflowers: Showy Locoweed, Oxytropis Splendens
Oxytropis in Flora of China @ efloras.org
Oxytropis Genus - Fabaceae Family
Oxytropis shennongjiaensis | International Plant Names Index
CalPhotos: Oxytropis lambertii; Lambert Crazyweed
Oxytropis lambertii Archives - Colorado Native Plant Society
EcoFlora - Oxytropis carpatica
Loco weeds: Oxytropis
Genus: Oxytropis (oxytrope): Go Botany
Oxytropis setosa - Useful Temperate Plants
UKnowledge - IGC Proceedings (2001-2021): Effect of Oxytropis Species Consumption on Sheep Forage Preference in Northeastern...
Oxytropis splendens Douglas ex Hook. GRIN-Global
Lady Bird Johnson Wildflower Center - The University of Texas at Austin
Oxytropis deflexa var. sericea | Encyclopedia of Puget Sound
Version 10.38 20 July 2010 - Scientific Names: Oxytropis karataviensis Pavlov.
Seed Starting Chronicles 2014 | North American Rock Garden Society
Molecular Phylogeny of Ten Species of Oxytropis Based on psbAtrnH Sequences
Tyria jacobaeae on Salvia : Photos, Diagrams & Topos : SummitPost
FLOREALPES : Fleurs des Alpes, de Montagne, de Provence, de Corse et des Pyrénées
IndexCat
Alpine Garden Club 2006 Seedlist
Biomarkers Search
Species Conservation Status | Idaho Fish and Game
The Euro+Med Plantbase Project
Species Conservation Status | Idaho Fish and Game
Collecting localities - Biodiversity of the Hengduan Mountains
Genus1
- Oxytropis is a genus of plants in the legume family. (wikipedia.org)
Borealis2
- Sticky Locoweed, formerly Oxytropis viscida , is part of the Oxytropis borealis complex, which consists of 4 variable varieties that are distinguished by flower color and size, compactness of the flower clusters, and some leaf characteristics. (minnesotawildflowers.info)
- Common name: Oxytropis viscidula, Viscid locoweed, Oxytropis borealis var. (plant-world-seeds.com)
Lambertii2
- Oxytropis lambertii Pursh is an accepted species name sensu FNA Ed. Comm. (canadensys.net)
- Oxytropis comes from oxys, "sharp," and tropis, "keel," in reference to the beaked flower petals, while the meaning of lambertii is unknown. (asu.edu)
Pursh1
- Oxytropis lamberti Pursh. (funet.fi)
Oreophila1
- These plots show the elevations and times of year where the plant Oxytropis oreophila has been observed. (wildflowersearch.org)
Sericea3
- Oxytropis sericea subsp. (gc.ca)
- Utilization of White Locoweed (Oxytropis sericea Nutt. (arizona.edu)
- Utilization studies conducted on a high mountain range determined the quantity and timing of white locoweed (Oxytropis sericea Nutt. (arizona.edu)
Campestris1
- Oxytropis campestris (L.) DC. (funet.fi)
Viscida1
- I think I see this species ( Oxytropis viscida in Flora of Alberta ) along the Bow River uplands ( http://nargs.org/smf/index.php?topic=694.msg9762#msg9762 ), although I need to lug the book along sometime and see if I can actually key it out. (nargs.org)
Species4
- This Oxytropis species is found across a vast expanses of North America. (nargs.org)
- What a lovely mix of colors in your photos.They are indeed Oxytropis and I agree they appear to be the same species. (nargs.org)
- Locoweeds are Astragalus and Oxytropis species that contain the toxic alkaloid swainsonine, causing widespread poisoning of livestock in Inner Mongolia. (usda.gov)
- Taxa (Astragalus, Oxytropis, Sphaerophysa, and Sophora species) suspected of causing locoism and/or poisoning in Inner Mongolia were surveyed for swainsonine and Undifilum, the fungal endophyte responsible for the production of swainsonine. (usda.gov)
Plants1
- Larkspur (Delphinium spp), loco weed (Oxytropis spp), halogeton (Halogeton glomeratus), lupine (Lupinus spp), deathcamas (Zegadenus spp), and horsebrush (Tetradymia spp) are the poisonous plants that are widely distributed in limited quantities throughout the area. (blm.gov)
Aragallus1
- Aragallus aven-nelsonii , Aragallus falcatus Greene, Aragallus involutus , Oxytropis bushii , Oxytropis plattensis Nutt. (asu.edu)
Sordida1
- Oxytropis sordida (Willd. (funet.fi)
Deflexa1
- Oxytropis deflexa var. (gc.ca)
Halleri1
- Oxytropis halleri commonly known as Mountain Milk-vetch is a Nationally Rare plant confined only to Scotland. (rbge.info)
Uralensis2
- ܥ ˥ 륢 / ʪ Ƽ?? Oxytropis uralensis. (mycomfy-shop.com)
- Oxytropis uralensis. (mycomfy-shop.com)
Lapponica3
- Oxytropis lapponica (Wahlenb. (funet.fi)
- Oxytropis lapponica is a herb with odd number of leaflets. (valleyofflowers.info)
- Following are some of the pictures of the Oxytropis lapponica,hope you will like them all. (valleyofflowers.info)
Splendens1
- Oxytropis splendens Dougl. (funet.fi)
Astragalus1
- The prominent keel makes one think of Oxytropis but maybe it is an Astragalus. (nargs.org)
Keel1
- Oxytropis has a prominent beaked keel, lacks stems (acaulesecnt), and has erect or ascending scapes (flower stalk). (asu.edu)
20231
- https://www.plantarium.ru/lang/en/page/image/id/695225.html (accessed on 8 Dec 2023). (plantarium.ru)
Image1
- 2021. Image of Oxytropis trichocalycina Bunge ex Boiss. (plantarium.ru)
Names3
- Scientific Names: Oxytropis mertensiana Turcz. (legumes-online.net)
- Scientific Names: Oxytropis dorogostajskyi Kuzen. (legumes-online.net)
- Scientific Names: Oxytropis tyttantha Gontsch. (legumes-online.net)
Distribution1
- Predictive spatial models of the distribution of Oxytropis triphylla (Pall. (bio-conferences.org)
Species9
- Locoweeds are members primarily of the genus Oxytropis, but only a few species in each genus cause locoism. (plant-life.org)
- A revision of the North American species of Oxytropis DC. (vurv.cz)
- Total DNA from 56 individual plants collected in 28 sample sites of 10 Oxytropis species in Inner Mongolia was extracted in this research, and the psbAtrnH sequence was amplied and sequenced. (kib.ac.cn)
- The investigation results indicated that psbAtrnH sequences provided the molecular evidences for the study of the phylogenetic relationships in the Oxytropis at subgenus, sects and species levels. (kib.ac.cn)
- Molecular Phylogeny of Ten Species of Oxytropis Based on psbAtrnH Sequences[J]. Plant Diversity, 2014, 36(03): 279-284. (kib.ac.cn)
- 2009. Molecular phylogeny of several species of Oxytropis DC. (kib.ac.cn)
- However, the results suggest that New World Oxytropis species did not evolve by a single adaptive radiation in the genus, but rather from different Old World lineages. (metu.edu.tr)
- Although the Oxytropis species did not show high genetic diversity, one subcluster of the genus was always distinctly separated in both trees. (metu.edu.tr)
- This subcluster was formed by the species Oxytropis engizekensis Duman & Vural and O. persica Boiss. (metu.edu.tr)
Genus Oxytropis1
- The keel is prolonged into a point or tooth or generally into a straight or curved beak, this is a distinctive feature of the genus Oxytropis. (plant-life.org)
Phylogenetic relationships1
- Phylogenetic relationships between Oxytropis DC. (metu.edu.tr)
Molecular phylogeny1
- 2011. Molecular phylogeny of Oxytropis DC. (kib.ac.cn)
Candolle1
- 140. Oxytropis Candolle, Astragalogia. (efloras.org)
Bunge1
- 15. Ochrocephalamines B-D, Three Alkaloids from Oxytropis ochrocephala Bunge. (nih.gov)
Sequences1
- In addition to our samples, 36 Oxytropis ITS and 6 Oxytropis matK sequences were retrieved from GenBank and included in the analysis. (metu.edu.tr)
Locoweeds1
- Oxytropis, or locoweeds, have flowers in usually quite compact clusters, on stalks that rise from the base of the plant. (plant-life.org)
Names1
- Scientific Names: Oxytropis karataviensis Pavlov. (legumes-online.net)
Samples1
- 3) The samples of Oxytropis glabra var. (kib.ac.cn)
Effect1
- 3. The effect of alkaloid from Oxytropis ochrocephala on growth inhibition and expression of PCNA and p53 in mice bearing H22 Hepatocellular Carcinoma. (nih.gov)