AnatomyDiseasesChemicals and DrugsPsychiatry and PsychologyPhenomena and ProcessesDisciplines and OccupationsAnthropology, Education, Sociology and Social PhenomenaHumanitiesInformation ScienceHealth CareGeographicals
Muscular DiseasesMovementPostureHuman BodyMusclesSubstance PMuscle, SkeletalMuscle ProteinsNeuromuscular DiseasesMuscular DystrophiesMuscular Dystrophy, DuchenneHospitals, VoluntaryMuscular Dystrophy, AnimalHospitals, ProprietaryMyotonic DystrophyMuscular Dystrophy, FacioscapulohumeralChromosomes, Human, Pair 4DystrophinMuscular Atrophy, SpinalSpinal Muscular Atrophies of ChildhoodSurvival of Motor Neuron 1 ProteinSMN Complex ProteinsSurvival of Motor Neuron 2 ProteinHuntington DiseaseAtrophyLos AngelesAmerican Heart AssociationCardiologyCongresses as TopicHeartMotor NeuronsMotor Neuron DiseaseSarcoglycansMuscular Dystrophies, Limb-GirdleCalpainDystroglycansBiological Ontologies
Muscular Diseases: Acquired, familial, and congenital disorders of SKELETAL MUSCLE and SMOOTH MUSCLE.Movement: The act, process, or result of passing from one place or position to another. It differs from LOCOMOTION in that locomotion is restricted to the passing of the whole body from one place to another, while movement encompasses both locomotion but also a change of the position of the whole body or any of its parts. Movement may be used with reference to humans, vertebrate and invertebrate animals, and microorganisms. Differentiate also from MOTOR ACTIVITY, movement associated with behavior.Posture: The position or attitude of the body.Human Body: The human being as a non-anatomical and non-zoological entity. The emphasis is on the philosophical or artistic treatment of the human being, and includes lay and social attitudes toward the body in history. (From J. Cassedy, NLM History of Medicine Division)Muscles: Contractile tissue that produces movement in animals.Substance P: An eleven-amino acid neurotransmitter that appears in both the central and peripheral nervous systems. It is involved in transmission of PAIN, causes rapid contractions of the gastrointestinal smooth muscle, and modulates inflammatory and immune responses.Muscle, Skeletal: A subtype of striated muscle, attached by TENDONS to the SKELETON. Skeletal muscles are innervated and their movement can be consciously controlled. They are also called voluntary muscles.Muscle Proteins: The protein constituents of muscle, the major ones being ACTINS and MYOSINS. More than a dozen accessory proteins exist including TROPONIN; TROPOMYOSIN; and DYSTROPHIN.Neuromuscular Diseases: A general term encompassing lower MOTOR NEURON DISEASE; PERIPHERAL NERVOUS SYSTEM DISEASES; and certain MUSCULAR DISEASES. Manifestations include MUSCLE WEAKNESS; FASCICULATION; muscle ATROPHY; SPASM; MYOKYMIA; MUSCLE HYPERTONIA, myalgias, and MUSCLE HYPOTONIA.Muscular Dystrophies: A heterogeneous group of inherited MYOPATHIES, characterized by wasting and weakness of the SKELETAL MUSCLE. They are categorized by the sites of MUSCLE WEAKNESS; AGE OF ONSET; and INHERITANCE PATTERNS.Muscular Dystrophy, Duchenne: An X-linked recessive muscle disease caused by an inability to synthesize DYSTROPHIN, which is involved with maintaining the integrity of the sarcolemma. Muscle fibers undergo a process that features degeneration and regeneration. Clinical manifestations include proximal weakness in the first few years of life, pseudohypertrophy, cardiomyopathy (see MYOCARDIAL DISEASES), and an increased incidence of impaired mentation. Becker muscular dystrophy is a closely related condition featuring a later onset of disease (usually adolescence) and a slowly progressive course. (Adams et al., Principles of Neurology, 6th ed, p1415)Hospitals, Voluntary: Private, not-for-profit hospitals that are autonomous, self-established, and self-supported.Muscular Dystrophy, AnimalHospitals, Proprietary: Hospitals owned and operated by a corporation or an individual that operate on a for-profit basis, also referred to as investor-owned hospitals.Myotonic Dystrophy: Neuromuscular disorder characterized by PROGRESSIVE MUSCULAR ATROPHY; MYOTONIA, and various multisystem atrophies. Mild INTELLECTUAL DISABILITY may also occur. Abnormal TRINUCLEOTIDE REPEAT EXPANSION in the 3' UNTRANSLATED REGIONS of DMPK PROTEIN gene is associated with Myotonic Dystrophy 1. DNA REPEAT EXPANSION of zinc finger protein-9 gene intron is associated with Myotonic Dystrophy 2.Muscular Dystrophy, Facioscapulohumeral: An autosomal dominant degenerative muscle disease characterized by slowly progressive weakness of the muscles of the face, upper-arm, and shoulder girdle. The onset of symptoms usually occurs in the first or second decade of life. Affected individuals usually present with impairment of upper extremity elevation. This tends to be followed by facial weakness, primarily involving the orbicularis oris and orbicularis oculi muscles. (Neuromuscul Disord 1997;7(1):55-62; Adams et al., Principles of Neurology, 6th ed, p1420)Chromosomes, Human, Pair 4: A specific pair of GROUP B CHROMOSOMES of the human chromosome classification.Dystrophin: A muscle protein localized in surface membranes which is the product of the Duchenne/Becker muscular dystrophy gene. Individuals with Duchenne muscular dystrophy usually lack dystrophin completely while those with Becker muscular dystrophy have dystrophin of an altered size. It shares features with other cytoskeletal proteins such as SPECTRIN and alpha-actinin but the precise function of dystrophin is not clear. One possible role might be to preserve the integrity and alignment of the plasma membrane to the myofibrils during muscle contraction and relaxation. MW 400 kDa.Muscular Atrophy, Spinal: A group of disorders marked by progressive degeneration of motor neurons in the spinal cord resulting in weakness and muscular atrophy, usually without evidence of injury to the corticospinal tracts. Diseases in this category include Werdnig-Hoffmann disease and later onset SPINAL MUSCULAR ATROPHIES OF CHILDHOOD, most of which are hereditary. (Adams et al., Principles of Neurology, 6th ed, p1089)Spinal Muscular Atrophies of Childhood: A group of recessively inherited diseases that feature progressive muscular atrophy and hypotonia. They are classified as type I (Werdnig-Hoffman disease), type II (intermediate form), and type III (Kugelberg-Welander disease). Type I is fatal in infancy, type II has a late infantile onset and is associated with survival into the second or third decade. Type III has its onset in childhood, and is slowly progressive. (J Med Genet 1996 Apr:33(4):281-3)Survival of Motor Neuron 1 Protein: A SMN complex protein that is essential for the function of the SMN protein complex. In humans the protein is encoded by a single gene found near the inversion telomere of a large inverted region of CHROMOSOME 5. Mutations in the gene coding for survival of motor neuron 1 protein may result in SPINAL MUSCULAR ATROPHIES OF CHILDHOOD.SMN Complex Proteins: A complex of proteins that assemble the SNRNP CORE PROTEINS into a core structure that surrounds a highly conserved RNA sequence found in SMALL NUCLEAR RNA. They are found localized in the GEMINI OF COILED BODIES and in the CYTOPLASM. The SMN complex is named after the Survival of Motor Neuron Complex Protein 1, which is a critical component of the complex.Survival of Motor Neuron 2 Protein: A SMN complex protein that is closely-related to SURVIVAL OF MOTOR NEURON 1 PROTEIN. In humans, the protein is encoded by an often duplicated gene found near the inversion centromere of a large inverted region of CHROMOSOME 5.Huntington Disease: A familial disorder inherited as an autosomal dominant trait and characterized by the onset of progressive CHOREA and DEMENTIA in the fourth or fifth decade of life. Common initial manifestations include paranoia; poor impulse control; DEPRESSION; HALLUCINATIONS; and DELUSIONS. Eventually intellectual impairment; loss of fine motor control; ATHETOSIS; and diffuse chorea involving axial and limb musculature develops, leading to a vegetative state within 10-15 years of disease onset. The juvenile variant has a more fulminant course including SEIZURES; ATAXIA; dementia; and chorea. (From Adams et al., Principles of Neurology, 6th ed, pp1060-4)Atrophy: Decrease in the size of a cell, tissue, organ, or multiple organs, associated with a variety of pathological conditions such as abnormal cellular changes, ischemia, malnutrition, or hormonal changes.Los AngelesAmerican Heart Association: A voluntary organization concerned with the prevention and treatment of heart and vascular diseases.Cardiology: The study of the heart, its physiology, and its functions.Congresses as Topic: Conferences, conventions or formal meetings usually attended by delegates representing a special field of interest.Heart: The hollow, muscular organ that maintains the circulation of the blood.Motor Neurons: Neurons which activate MUSCLE CELLS.Motor Neuron Disease: Diseases characterized by a selective degeneration of the motor neurons of the spinal cord, brainstem, or motor cortex. Clinical subtypes are distinguished by the major site of degeneration. In AMYOTROPHIC LATERAL SCLEROSIS there is involvement of upper, lower, and brainstem motor neurons. In progressive muscular atrophy and related syndromes (see MUSCULAR ATROPHY, SPINAL) the motor neurons in the spinal cord are primarily affected. With progressive bulbar palsy (BULBAR PALSY, PROGRESSIVE), the initial degeneration occurs in the brainstem. In primary lateral sclerosis, the cortical neurons are affected in isolation. (Adams et al., Principles of Neurology, 6th ed, p1089)Sarcoglycans: A family of transmembrane dystrophin-associated proteins that play a role in the membrane association of the DYSTROPHIN-ASSOCIATED PROTEIN COMPLEX.Muscular Dystrophies, Limb-Girdle: A heterogenous group of inherited muscular dystrophy that can be autosomal dominant or autosomal recessive. There are many forms (called LGMDs) involving genes encoding muscle membrane proteins such as the sarcoglycan (SARCOGLYCANS) complex that interacts with DYSTROPHIN. The disease is characterized by progressing wasting and weakness of the proximal muscles of arms and legs around the HIPS and SHOULDERS (the pelvic and shoulder girdles).Calpain: Cysteine proteinase found in many tissues. Hydrolyzes a variety of endogenous proteins including NEUROPEPTIDES; CYTOSKELETAL PROTEINS; proteins from SMOOTH MUSCLE; CARDIAC MUSCLE; liver; platelets; and erythrocytes. Two subclasses having high and low calcium sensitivity are known. Removes Z-discs and M-lines from myofibrils. Activates phosphorylase kinase and cyclic nucleotide-independent protein kinase. This enzyme was formerly listed as EC 3.4.22.4.Dystroglycans: Dystrophin-associated proteins that play role in the formation of a transmembrane link between laminin-2 and DYSTROPHIN. Both the alpha and the beta subtypes of dystroglycan originate via POST-TRANSLATIONAL PROTEIN PROCESSING of a single precursor protein.Biological Ontologies: Structured vocabularies describing concepts from the fields of biology and relationships between concepts.

Toxic oil syndrome mortality: the first 13 years. (1/1572)

BACKGROUND: The toxic oil syndrome (TOS) epidemic that occurred in Spain in the spring of 1981 caused approximately 20000 cases of a new illness. Overall mortality and mortality by cause in this cohort through 1994 are described for the first time in this report. METHODS: We contacted, via mail or telephone, almost every living member of the cohort and family members of those who were known to have died in order to identify all deaths from 1 May 1981 through 31 December 1994. Cause of death data were collected from death certificates and underlying causes of death were coded using the International Classification of Diseases, 9th Revision. RESULTS: We identified 1663 deaths between 1 May 1981 and 31 December 1994 among 19 754 TOS cohort members, for a crude mortality rate of 8.4%. Mortality was highest during 1981, with a standardized mortality ratio (SMR) of 4.92 (95% confidence interval [CI]: 4.39-5.50) compared with the Spanish population as a whole. The highest SMR, (20.41, 95% CI: 15.97-25.71) was seen among women aged 20-39 years during the period from 1 May 1981 through 31 December 1982. Women <40 years old, who were affected by TOS , were at greater risk for death in most time periods than their unaffected peers, while older women and men were not. Over the follow-up period, mortality of the cohort was less than expected when compared with mortality of the general Spanish population, or with mortality of the population of the 14 provinces where the epidemic occurred. We also found that, except for deaths attributed to external causes including TOS and deaths due to pulmonary hypertension, all causes of death were decreased in TOS patients compared to the Spanish population. The most frequent underlying causes of death were TOS, 350 (21.1%); circulatory disorders, 536 (32.3%); and malignancies, 310 (18.7%). CONCLUSIONS: We conclude that while on average people affected by toxic oil syndrome are not at greater risk for death over the 13-year study period than any of the comparison groups, women <40 years old were at greater risk of death.  (+info)

Wasting of the small hand muscles in upper and mid-cervical cord lesions. (2/1572)

Four patients are described with destructive rheumatoid arthritis of the cervical spine and neurogenic wasting of forearm and hand muscles. The pathological connection is not immediately obvious, but a relationship between these two observations is described here with clinical, radiological, electrophysiological and necropsy findings. Compression of the anterior spinal artery at upper and mid-cervical levels is demonstrated to be the likely cause of changes lower in the spinal cord. These are shown to be due to the resulting ischaemia of the anterior part of the lower cervical spinal cord, with degeneration of the neurones innervating the forearm and hand muscles. These findings favour external compression of the anterior spinal artery leading to ischaemia in a watershed area as the likeliest explanation for this otherwise inappropriate and bizarre phenomenon.  (+info)

Obturator internus muscle abscess in children: report of seven cases and review. (3/1572)

Obturator internus muscle (OIM) abscess is an uncommon entity often mistaken for septic arthritis of the hip. We describe seven children with OIM abscess and review seven previously reported cases. The most common presenting symptoms were hip or thigh pain (14 patients), fever (13), and limp (13). The hip was flexed, abducted, and externally rotated in 11 patients. Magnetic resonance imaging and computed tomography (CT) were diagnostic for OIM abscess in the 14 patients. Associated abscesses were located in the obturator externus muscle (5 patients), psoas muscle (2), and iliac muscle (1). The etiologic agents were Staphylococcus aureus (8 patients), Streptococcus pyogenes (2), Neisseria gonorrhoeae (2), and Enterococcus faecalis (1). Three patients underwent CT-guided percutaneous drainage, and three had surgical drainage. Three patients had ischial osteomyelitis in addition to OIM abscess. The 11 children with uncomplicated OIM abscess were treated for a median of 28 days. All patients had an uneventful recovery.  (+info)

Autosomal dominant myopathy with proximal weakness and early respiratory muscle involvement maps to chromosome 2q. (4/1572)

Two Swedish families with autosomal dominant myopathy, who also had proximal weakness, early respiratory failure, and characteristic cytoplasmic bodies in the affected muscle biopsies, were screened for linkage by means of the human genome screening set (Cooperative Human Linkage Center Human Screening Set/Weber version 6). Most chromosome regions were completely excluded by linkage analysis (LOD score <-2). Linkage to the chromosomal region 2q24-q31 was established. A maximum combined two-point LOD score of 4.87 at a recombination fraction of 0 was obtained with marker D2S1245. Haplotype analysis indicated that the gene responsible for the disease is likely to be located in the 17-cM region between markers D2S2384 and D2S364. The affected individuals from these two families share an identical haplotype, which suggests a common origin.  (+info)

Weakness associated with the pathological presence of lipid in skeletal muscle: a detailed study of a patient with carnitine deficiencey. (5/1572)

A patient with muscular weakness demonstrating pathological lipid accumulation and abnormal mitochondria in skeletal muscle has been studied. The lipid accumulation and mitochondrial changes are thought to be related to the established deficiency of carnitine in this patient's muscle. The symptoms of muscular weakness associated with lipid accumulation in the skeletal muscle in the absence of complaint of muscle cramps or myglobinuria are thought to be diagnostic of carnitine deficiency. The failure of the sarcoplasmic reticulum to accumulate Ca2+ is discussed. The patient's strength responded dramatically when propranolol was added to his steroid therapy.  (+info)

Measurements of muscle strength and performance in children with normal and diseased muscle. (6/1572)

A study has been made of two simple means of measuring muscle power in children with normal and diseased muscle. In one the length of time that the leg and the head could be held at 45 degrees above the horizontal was measured with the child supine. In the second, measurements were made of the isometric strength of six muscle groups with the newly developed Hammersmith Myometer. In the timed performance tests only 5 (8%) of a group of 61 children known to have muscle disease achieved the minimum expected values for their ages. Myometer readings of the isometric power of the children with muscle disease also have values which were below those of a comparable group of normal children. The reproducibility of muscle strength measurements in young children has been shown to be good, whereas the timed performance tests, though able to differentiate normal children from children with muscle disease, did not show sufficient reporducibility for this test to be recommended for sequential measurements.  (+info)

Calcific myonecrosis. (7/1572)

Calcific myonecrosis is a rare and late sequela of compartment syndrome, which becomes symptomatic years after the initial trauma. We diagnosed this condition in a 64-year old man, 42 years after he sustained a shot-gun wound to the right lower leg. Total excision of a peripherally calcified, cystic mass, continuous with the anterior tibial muscle belly resulted in complete resolution of symptoms. Consideration of the diagnosis is warranted in patients with a history of major injury who develop a soft tissue mass in the traumatized compartment. The treatment of choice is marginal excision.  (+info)

The prevalence and CT appearance of the levator claviculae muscle: a normal variant not to be mistaken for an abnormality. (8/1572)

BACKGROUND AND PURPOSE: The levator claviculae muscle is an infrequently recognized variant in humans, occurring in 2% to 3% of the population, and has rarely been reported in the radiologic or anatomic literature. The importance of this muscle to radiologists is in distinguishing it from an abnormality; most commonly, cervical adenopathy. After discovering this muscle on the CT scans of two patients during routine clinical examinations, we conducted a study to determine the prevalence and appearance of the muscle on CT studies. METHODS: We evaluated 300 CT scans that adequately depicted the expected location of the muscle. The most superior level in which the muscle could be identified and the apparent location of insertion on the clavicle were recorded for all subjects in whom the muscle was detected. RESULTS: Seven levator claviculae muscles were identified in six subjects (2%). It was bilateral in one, on the left in four, and on the right in one. It was identified up to the level of the transverse process of C3 in all cases. The insertion was the middle third of the clavicle for two muscles and the lateral third of the clavicle for the remaining five muscles. CONCLUSION: Because the levator claviculae muscle will most likely be encountered during a radiologist's career, it is important to recognize this muscle as a variant and not as an abnormality.  (+info)

Statin-Induced MyopathyStatin-Induced Myopathy
This page includes the following topics and synonyms: Statin-Induced Myopathy, Rhabdomyolysis due to Statin, Statin-Induced Myalgias, Myositis due to Statins, Statin Myopathy.
Diagnostic Criteria of Stevens et al for Critical Illness Myopathy (CIM) | Medicalalgorithms.comDiagnostic Criteria of Stevens et al for Critical Illness Myopathy (CIM) | Medicalalgorithms.com
Stevens et al listed criteria for the diagnosis of Critical Illness Myopathy (CIM) in a patient with intensive care unit-acquired weakness. The authors are from the Johns Hopkins University, Uniformed Services University of Health Sciences, Ohio State University, Centre Hospitalier de Poissy-Saint-Germain and University of Versailles Saint-Quentin en Yvelines Garches.
Clinical Trial of CoQ10 for Mild-to-Moderate Statin-Associated Muscle Symptoms - Full Text View - ClinicalTrials.govClinical Trial of CoQ10 for Mild-to-Moderate Statin-Associated Muscle Symptoms - Full Text View - ClinicalTrials.gov
This study will recruit individuals who have had muscle symptoms while using a statin. During the first part of the study, volunteers will be given a statin medication to see if their muscle symptoms return. Those who experience muscle symptoms on this statin rechallenge will be invited to continue in the second part of the trial, in which participants will be randomly assigned to receive either CoQ10 or a placebo (sugar pill) to take with statin medication. We hypothesize that those who receive CoQ10 will experience an improvement in their muscle symptoms compared to those who receive placebo, and, secondarily, that those who receive CoQ10 will be more likely to continue taking the statin medication ...
The Association of Genetic Polymorphisms With Statin-Induced Myopathy. - Full Text View - ClinicalTrials.govThe Association of Genetic Polymorphisms With Statin-Induced Myopathy. - Full Text View - ClinicalTrials.gov
Jisun Oh, et al. Genetic determinants of statin intolerance. Lipid in Health and Disease 2007;6(7). Wei Zhang, et al. Role of BCRP 421C？A polymorphism on rosuvastatin pharmacokinetics in healthy Chinese males. Clinica Chimica Acta 2006;373:99-103. Mikko Niemi, et al. Association of genetic polymorphism in ABCC2 with hepatic multidrug resistance-associated protein 2 expression and pravastatin pharmacokinetics. Pharmacogenetics and Genomics 2006;16:801-808. Andre BM, et al. Association of polymorphism in the cytochrome CYP2D6 and the efficacy and tolerability of simvastatin. Clin Pharmacol Ther 2001;70:546-551. K. Morimoto, et al. OATP-C(OATPO1B1)15 IS ASSOCIATED WITH LESTEROLEMIC PATIENTS. CLINICAL PHARMACOLOGY THERAPEUTICS 2005;77(2). K. Morimoto, et al. CANDIDATE OF GENETIC MARKERS FOR STATIN-INDUCED MYOPATHY IN JAPANESE PATIENTS WITH HYPERCHOLESTEROLEMIA. Drug Metabolism Reviews 2005;37(4):345 ...
Progressive myopathy and defects in the maintenance of myotendinous junctions in mice that lack talin 1 in skeletal muscle |...Progressive myopathy and defects in the maintenance of myotendinous junctions in mice that lack talin 1 in skeletal muscle |...
We show here that talin 1 is crucial for the maintenance of integrin attachment sites at MTJs. Tln1HSA-CREko mice were viable and fertile, but suffered from a progressive myopathy. Whereas integrins and some of their effectors such as FAK, Ilk and vinculin still were localized to muscle attachment sites at costameres and MTJs, MTJs showed structural abnormalities. Defects in the ultrastructure of MTJs, such as decreased interdigitations of muscle and tendon and retraction of myofilaments from electron-dense plaques at the plasma membrane, indicate that in the absence of talin 1 the mechanical connection of actin filaments and integrins at the MTJ was compromised. By contrast, sarcolemmal integrity was largely maintained. Defects in skeletal muscle were prominent in 6- to 7-month-old mice, and were only occasionally noted in 1- to 2-month-old animals, suggesting that the defects were caused by mechanical failure of MTJs under duress. In agreement with this finding, isolated muscle fibers from 7 ...
Altered sodium channel-protein associations in critical illness myopathyAltered sodium channel-protein associations in critical illness myopathy
PubMed Central Canada (PMC Canada) provides free access to a stable and permanent online digital archive of full-text, peer-reviewed health and life sciences research publications. It builds on PubMed Central (PMC), the U.S. National Institutes of Health (NIH) free digital archive of biomedical and life sciences journal literature and is a member of the broader PMC International (PMCI) network of e-repositories.
Notes from Dr. RW: Statin myopathyNotes from Dr. RW: Statin myopathy
This review emphasizes that statin myopathy is heterogeneous, and discussed the newly appreciated necrotizing autoimmune variant which may persist after statin removal. ...
tight muscles - Symptoms, Treatments and Resources for tight musclestight muscles - Symptoms, Treatments and Resources for tight muscles
tight muscles - MedHelps tight muscles Center for Information, Symptoms, Resources, Treatments and Tools for tight muscles. Find tight muscles information, treatments for tight muscles and tight muscles symptoms.
The Relation of Alcoholic Myopathy to Cardiomyopathy | Annals of Internal Medicine | American College of PhysiciansThe Relation of Alcoholic Myopathy to Cardiomyopathy | Annals of Internal Medicine | American College of Physicians
Results:. Alcoholic patients with cardiomyopathy had less muscle strength than did alcoholic patients with normal cardiac function, patients with idiopathic dilated cardiomyopathy, and patients with coronary heart disease (all P , 0.01). Among alcoholic patients with cardiomyopathy, 20 of 24 (83%) had histologic findings of skeletal myopathy compared with 1 of 24 (4%) alcoholic patients with normal cardiac function (P , 0.001). Interstitial fibrosis occurred in all cardiac biopsy specimens, hypertrophy of the myocytes occurred in 95%, and myocytolysis occurred in 83%. Those patients with more severe cellular hypertrophy and interstitial fibrosis of the myocardium had a greater decrease in deltoid muscle strength and had worse histologic myopathy. ...
Hand symptoms and Long thin fingers and Muscle symptoms and Ocular weakness and Skin texture changes and Tooth deformity -...Hand symptoms and Long thin fingers and Muscle symptoms and Ocular weakness and Skin texture changes and Tooth deformity -...
List of causes of Hand symptoms and Long thin fingers and Muscle symptoms and Ocular weakness and Skin texture changes and Tooth deformity, alternative diagnoses, rare causes, misdiagnoses, patient stories, and much more.
Muscle symptoms and Oral pain and Face symptoms - Symptom Checker - check medical symptoms at RightDiagnosisMuscle symptoms and Oral pain and Face symptoms - Symptom Checker - check medical symptoms at RightDiagnosis
List of 43 causes of Muscle symptoms and Oral pain and Face symptoms, alternative diagnoses, rare causes, misdiagnoses, patient stories, and much more.
Annals of Indian Academy of Neurology: Table of ContentsAnnals of Indian Academy of Neurology: Table of Contents
Thirteen cases of vacuolar myopathy (6 males, 7 females), with age range of 4 months to 22 years and diagnosed over a period from 1986 to 1999, could be categorized into acid maltase deficiency (AMD) (n=6), carnitine deficiency (CD) (n=5), and mitochondria-lipid-glycogen myopathy (MLGM) (n=2), cases of AMD presented as floppy infants with reparatory infection, while cases of carnitine deficiency presented with progressive motor weakness with normal initial milestones. Delayed motor milestones and proximal muscle weakness was the presenting complaints in MLGM. The diagnosis in all these cases was established based on the morphological findings on muscle biopsy, namely demonstration of PAS positive material within the vacuoles in AMD, ragged red fibers, vacuoles containing neutral fats and abnormal mitochondria in CD. MLGM was characterized by the presence of PAS positive material and neutral fat. The diagnosis was confirmed by identification of abnormal mitochondria under electron microscope. The ...
A Trigger for Muscular DiseasesA Trigger for Muscular Diseases
Various muscular diseases are associated with changes in the elasticity of the protein titin, but whether these changes are a cause or an effect of disease has been unclear. Researchers help solve this
Seeing an MFM on Thursday! - Welcome to the Preeclampsia FoundationSeeing an MFM on Thursday! - Welcome to the Preeclampsia Foundation
Honestly, based purely on my observations here, the MFM situation with the first doctor is a little unusual. Its actually pretty normal for MFMs to either be a primary doctor by themselves with no regular OB in the picture at all, or MFMs that co-manage with the OB doing the typical OB stuff while the MFM keeps an extra eye on things and directs the plan. Many MFMs dont deliver at all. My personal MFM situation is my MFM can do either, she is comfortable being a primary and delivering her patients, or co-managing (although she confided to me that she will not co-manage with just any old OB, it has to be someone she feels is a quality OB.) Ive not heard of an MFM that co-manages but takes the delivery except in emergencies...I dont really understand the OBs role with the first MFM. In any case, as long as you have an OB that you like and trust to deliver you, as far as my non-expert understanding, theres no reason they cant deliver you whether your pregnancy goes smoothly or not unless ...
Drugs Used to Treat Bone and Muscle Disorders - Special Pet Topics - Merck Veterinary ManualDrugs Used to Treat Bone and Muscle Disorders - Special Pet Topics - Merck Veterinary Manual
Learn about the veterinary topic of Drugs Used to Treat Bone and Muscle Disorders. Find specific details on this topic and related topics from the Merck Vet Manual.
NMJ and muscle disorders Flashcards by Hiba Alhasso | BrainscapeNMJ and muscle disorders Flashcards by Hiba Alhasso | Brainscape
Study NMJ and muscle disorders flashcards from Hiba Alhasso's class online, or in Brainscape's iPhone or Android app. ✓ Learn faster with spaced repetition.
Mahamasha thailam-Ayurvedic Oil for Neurological & Muscular Diseases | Ayurvedic ProductsMahamasha thailam-Ayurvedic Oil for Neurological & Muscular Diseases | Ayurvedic Products
Contact Pure Herbal Ayurved Clinic to buy Ayurvedic Mahamasha thailam Oil for Muscular dystrophy, Joint stiffness, Frozen shoulder, Locked jaw in Melbourne, Australia.
European outbreaks of atypical myopathy in grazing horses (2006-2009): Determination of indicators for risk and prognostic...European outbreaks of atypical myopathy in grazing horses (2006-2009): Determination of indicators for risk and prognostic...
Reasons for performing study: Appropriate management of atypical myopathy (AM) requires the establishment of an accurate diagnosis and prognosis. Furthermore, preventive measures to avoid AM need to be refined.. Objectives: The aims of the study were as follows: 1) to improve the diagnosis of AM; 2) to identify prognostic predictors; and 3) to refine recommended preventive measures based on indicators of risk factors.. Methods: An exploratory analysis of cases in Europe between 2006 and 2009 reported to the Atypical Myopathy Alert Group was conducted. Based on clinical data, reported cases were allocated into 2 groups: confirmed or highly probable AM (AM group; further divided into survivors and nonsurvivors); and cases with a low probability of having AM or with another final diagnosis (non-AM group). Using Welchs test and odds ratios corrected for multiple comparisons, the AM vs. non-AM groups were compared to identify indicators for diagnosis and risk factors, and survivors vs. nonsurvivors ...
Myopathy, Distal 2 Symptoms, Diagnosis, Treatments and Causes - RightDiagnosis.comMyopathy, Distal 2 Symptoms, Diagnosis, Treatments and Causes - RightDiagnosis.com
Myopathy, Distal 2 information including symptoms, diagnosis, misdiagnosis, treatment, causes, patient stories, videos, forums, prevention, and prognosis.
MyopathyMyopathy
Create healthcare diagrams like this example called Myopathy in minutes with SmartDraw. SmartDraw includes 1000s of professional healthcare and anatomy chart templates that you can modify and make your own.
Critical illness polyneuropathy-1 - Critical illness polyneuropathy Critical illness polyneuropathy From = Critical care...Critical illness polyneuropathy-1 - Critical illness polyneuropathy Critical illness polyneuropathy From = Critical care...
View Notes - Critical illness polyneuropathy-1 from STEP 1 at Montgomery College. Critical illness polyneuropathy Critical illness polyneuropathy From = Critical care medicine 2002 ,8 , 302-310
ICD-10 Diagnosis Code G62.81 Critical illness polyneuropathyICD-10 Diagnosis Code G62.81 Critical illness polyneuropathy
Diagnosis Code G62.81 information, including descriptions, synonyms, code edits, diagnostic related groups, ICD-9 conversion and references to the diseases index.
Notes from Dr. RW: Neurologic complications of critical illnessNotes from Dr. RW: Neurologic complications of critical illness
Critical illness polyneuropathy (CIP), critical illness myopathy (CIM) and septic encephalopathy are under recognized complications of sepsis, multiorgan failure and prolonged mechanical ventilation. A review in the May issue of Chest focuses on CIP and CIM (often overlapping and sometimes lumped as ICU associated weakness). An editorial in the same issue discusses CIP and CIM and mentions the related entity septic encephalopathy, a state of impaired consciousness and cognition associated with sepsis ...
Similar papers for Myotilinopathy: refining the clinical and myopathological phenotype. - Semantic ScholarSimilar papers for Myotilinopathy: refining the clinical and myopathological phenotype. - Semantic Scholar
Mutations in myotilin gene (MYOT) have been associated with variable syndromes including limb girdle muscular dystrophy type 1A (LGMD1A) and a subgroup of myofibrillar myopathy (MFM/MYOT). We studied six Spanish patients from three unrelated kindreds and seven patients without family history. Three previously reported and two novel disease-associated MYOT mutations were identified in this group of patients. The disease is characterized by the onset at the age of 42-77 years with muscle weakness initially in distal or proximal leg muscles, eventually spreading to other muscle groups of the lower and upper extremities. Associated signs of cardiomyopathy, respiratory failure and peripheral neuropathy are present in a fraction of patients. Myopathological features of focal myofibrillar destruction resulting in intracytoplasmic deposits, strongly immunoreactive to myotilin, multiple rimmed and centrally or subsarcolemmally located non-rimmed vacuoles and streaming Z-lines, were observed in each patient
Wing-N-Wave Labradors Skeletal Muscle MyopathyWing-N-Wave Labradors Skeletal Muscle Myopathy
Muscle weakness, abnormal gait and posture, and a decrease in tolerance to exercise are the most common symptoms affecting Labradors with HMLR. On the average, onset of symptoms usually occur at 3-4 months of age, however, some dogs demonstrate symptoms as early as 6-8 weeks or as late as 6-7 months of age. Abnormalities in gait and posture include a short, stilted stride, bunny-hopping, low head posture and an arched back. These symptoms become more obvious as the exercise continues and the dog tires or if the dog is exposed to cold weather. Eventually, if not allowed to rest or to keep warm, the dog may temporarily collapse. Rest improves symptoms but follow-up exercise quickly brings on a relapse. Other signs of HMLR include abnormalities of the joints including splay-foot, cow-hocking and hip dysplasia. Additionally, as the disease progresses, atrophy of the muscles in the limbs and head becomes apparent.. ...
Polysaccharide Storage Myopathy (PSSM): What should I feed?Polysaccharide Storage Myopathy (PSSM): What should I feed?
Feeds that are high in starch, such as sweet feed, maize, wheat, oats, barley, and molasses, appear to facilitate the development of type 1 and type 2 PSSM. That is why these ingredients should be avoided for horses that have PSSM. Extra calories can be provided in the form of fat (oil) for performance horses that are prone to PSSM. An important part of the management of PSSM horses is daily exercise. This enhances glucose utilization, and improves energy metabolism in skeletal muscle. If only the diet is changed, researchers have found that approximately 50% of horses improve. If both diet and exercise are altered, then 90% of horses have no or few episodes of tying-up. An old theory about tying-up is that it is due to too much lactic acid in the muscle. Many exercise studies have proven that this is absolutely not the case with PSSM.. The unique feature of PSSM is that the muscle cells in PSSM horses remove sugar from the blood stream and transported into their muscle at a faster rate, and ...
Assessment of mitochondrial oxidative phosphorylation in patient muscle biopsies, lymphoblasts, and transmitochondrial cell...Assessment of mitochondrial oxidative phosphorylation in patient muscle biopsies, lymphoblasts, and transmitochondrial cell...
Assessment of mitochondrial oxidative phosphorylation in patient muscle biopsies, lymphoblasts, and transmitochondrial cell lines
What are the probable congenital myopathies?What are the probable congenital myopathies?
Probable congenital myopathies Fingerprint body myopathy: Patients present with hypotonia from infancy, proximal muscle weakness, and a delay in attaining motor milestones. Weakness progresses slowl... more
Statin-Associated Autoimmune Myopathy | Iranian Neurological AssociationStatin-Associated Autoimmune Myopathy | Iranian Neurological Association
The use of statins in neurocardiovascular conditions has widely increased over the decades. Based on extension of indications major side effects of statin therapy became evident. In 1 of 10,000 treated persons per year, statins cause toxic muscle weakness and creatine kinase (CK) level elevation. A novel finding is that in some patients the statin-induced myopathy is caused by an autoimmune-mediated treatable myopathy by the presence of autoantibodies against 3-hydroxy-3-methylglutaryl coenzyme A (HMG-CoA) reductase (anti-HMGCR). At present its frequency is 2 to 3 in 100.000 patients treated with statins. This neuromuscular phenotype encompasses a proximal symmetric weakness, and a persisting more than 10 times the upper limit of the normal range elevated CK levels. EMG presents an active myopathy with an increased spontaneous activity while muscle MRI shows muscle edema. Muscle biopsy reveals myofiber necrosis and regeneration with limited inflammation, analogous to the immune-mediated ...
Credible Evidence: Anthony Colpo answers a question on Lipitor etc.Credible Evidence: Anthony Colpo answers a question on Lipitor etc.
A study published in the October-November-December 2009 issue of Primary Care Cardiovascular Journal, showed statin-induced myopathy is far more common than previously claimed by drug companies and health officials. Researchers analysed the patient records of one 8,000 patient practice and found only one recorded case of muscle symptoms in a patient taking statins. But after questioning 96 randomly selected statin-using patients from the practice, they identified 19 cases of potential muscle damage[2]. Grab a calculator and check the percentage difference between 1:8000 and 19:96, and youll have some idea of just how massively underreported statin side effects are ...
Anew mitochondria-related disease showing myopathy with episodic hyper-creatine kinase-emia.Anew mitochondria-related disease showing myopathy with episodic hyper-creatine kinase-emia.
ORIGINAL ARTICLE A New Mitochondria-Related Disease Showing Myopathy with Episodic Hyper-creatine Kinase-emia Yuji Okamoto, MD, PhD,1 Itsuro Higuchi, MD,1 Yusuke Sakiyama, MD,1 Shoko Tokunaga, MD,1 Osamu Watanabe, MD, PhD,1 Kimiyoshi Arimura, MD,2 Masanori Nakagawa, MD,3 and Hiroshi Takashima, MD, PhD1 Objective: To elucidate the relationship between mitochondrial DNA (mtDNA) alterations and a mitochondrial disease with a distinct combination of characteristic symptoms, namely episodic hyper-creatine kinase (CK)-emia and mild myopathy. Methods: We selected 9 patients with mtDNA np8291 alteration from 586 patients suspected to have a mitochondrial disease, and assessed them clinically, pathologically, and genetically. These 9 patients had undiagnosed mitochondrial myopathy with episodic hyper-CK-emia, all showing similar symptoms and progression. Results: Patients had mild muscle weakness and episodic hyper-CK-emia triggered by infections or drugs. Five of 9 patients were initially diagnosed with ...
Sensitivity of Troponin - American College of CardiologySensitivity of Troponin - American College of Cardiology
These 4 cases demonstrate that there proteins expressed in diseased skeletal that are detected by antibodies in the fourth generation and high sensitivity cTnT assays. Although sub-clinical cardiac pathology leading to cTnT elevation cannot be totally excluded on the basis of the study, as the authors point out, this is unlikely. These patients had no apparent heart disease and had normal cTnI levels. These findings are consistent with other studies that describe an elevation of cTnT with normal cTnI in patients with myopathies.(8, 9) This provocative study, however, leads to more questions than answers. What is the prevalence of cTnT elevation in patients with skeletal muscle disease? Are there certain skeletal muscle diseases that more commonly have an elevation in cTnT? Will elevation of cTnT in this setting confound the diagnosis of acute coronary syndrome in a significant number of patients that present to Emergency Departments? Further studies are needed to address these issues.. What are ...
Accumulation of Abnormal Mitochondria Causes & Reasons - SymptomaAccumulation of Abnormal Mitochondria Causes & Reasons - Symptoma
Accumulation of Abnormal Mitochondria Symptom Checker: Possible causes include Rectal Biopsy. Check the full list of possible causes and conditions now! Talk to our Chatbot to narrow down your search.
Myofibrillar Myopathy And Weight LossMyofibrillar Myopathy And Weight Loss
The second goal is to determine the cause of the myopathy. The episodic disorders are characterized by acute loss of strength that can return to normal within. Cytoplasmic body neuromyopathy presenting as respiratory failure and weight loss. for the tensile strength and integrity of myofibrils but not tor myogenic commitment, Muscle fibres are composed of myofibrils, for the development and. a so-called myofibrillar myopathy the myofibrils disintegrate in certain. The heart is more affected by the disease than previously thought, which cause sudden cardiac death. Your Stools Reveal Whether You Can Lose Weight.
Symptoms and signs of HyperthyroidismSymptoms and signs of Hyperthyroidism
Hyperthyroidism is often clinically obvious but treatment should never be instituted without biochemical confirmation. Differentiation of the mild case from anxiety states may be difficult; useful positive clinical markers are eye signs, a diffuse goitre, proximal myopathy and wasting. The hyperdynamic circulation with warm peripheries seen with hyperthyroidism can be contrasted with the clammy hands of anxiety. Hyperthyroidism. ...
Linkage to 18qter differentiates two clinically overlapping syndromes: congenital cataracts-facial dysmorphism-neuropathy ...Linkage to 18qter differentiates two clinically overlapping syndromes: congenital cataracts-facial dysmorphism-neuropathy ...
Facial dysmorphism described in the CCFDN seems to become more obvious during late childhood and appears to be more prominent in males than in females.1 Indeed, in family 2, the male patient was the only affected person presenting dysmorphic features.. Concerning ocular abnormalities, cataracts in MSS are usually congenital but may also occur during childhood. No other developmental abnormality of the eye has been reported in MSS, as opposed to microcornea which appears to be a major clinical and probably a diagnostic criterion of CCFDN.. Moreover MSS and CCFDN syndromes differ regarding myopathic changes. Progressive muscle weakness and muscle atrophy were reported to be among the cardinal signs of MSS.12 Most muscle biopsy reports have described myopathic changes including variation in muscle fibre size, rimmed vacuoles, necrotic and regenerative fibres, numerous internal nuclei, nuclear degeneration, endomysial fibrosis, and fatty replacement.12-17 Sewry et al13 suggested that a unique dense ...
GNE Myopathy and Cell Apoptosis: A Comparative Mutation Analysis | SpringerLinkGNE Myopathy and Cell Apoptosis: A Comparative Mutation Analysis | SpringerLink
In a number of genetic disorders such as GNE myopathy, it is not clear how mutations in target genes result in disease phenotype. GNE myopathy is a progres
Search Results - SPS - Specialist Pharmacy Service - The first stop for professional medicines adviceSearch Results - SPS - Specialist Pharmacy Service - The first stop for professional medicines advice
This updated Medicines Q&A evaluates the available evidence on the use of coenzyme Q10 supplementation to reduce the risk of statin-induced myopathy. There is some… ...
Severe muscle stiffness, pain, headaches, fatigue, & jaw tightness, spasms - Muscle Symptoms - Symptom | Our HealthSevere muscle stiffness, pain, headaches, fatigue, & jaw tightness, spasms - Muscle Symptoms - Symptom | Our Health
Hi. Im trying to make the rounds and post my experience with similar symptoms that you described in hopes of helping those suffering from what I was recently suffering from. I am a 30 year old mother of two who is active and involved with raising my kids while my husband works. Just a month ago I started to notice SEVERE tension in my neck, back, shoulders, and head. I had some spasms throughout my chest area which was also very sore and sensitive. My skin was slightly tingly and I had some numbness in my hands and toes. My inner ear area was also beginning to get "sensitive". I assumed that my dislocated C1 & C2 from a car accident 18 years ago was the cause even though it never did anything like this over an 18 year period. I scheduled multiple appointments with my primary doctor, cardiologist, and neurologist. I just knew I would be told I was either suffering from chronic fatigue syndrome, fibromyalgia, heart disease, lupus, or some form of cancer. I honestly felt that I would be dying in a ...
Myofibrillar Myopathy disease: Malacards - Research Articles, Drugs, Genes, Clinical TrialsMyofibrillar Myopathy disease: Malacards - Research Articles, Drugs, Genes, Clinical Trials
NIH Rare Diseases : 50 myofibrillar myopathies (mfm) are a group of neuromuscular disorders characterized by slowly progressive weakness that can involve both proximal muscles (such as hips and shoulders) and distal muscles (those farther away from the trunk). some affected individuals also experience sensory symptoms, muscle stiffness, aching, or cramps. peripheral neuropathy or cardiomyopathy may also be present. most people with mfm begin to develop muscle weakness in mid-adulthood, but features of the condition can appear anytime between infancy and late adulthood. it may be caused by mutations in any of several genes, including des, cryab, myot, ldb3, flnc, and bag3; the signs and symptoms of mfm can vary widely depending on the conditions genetic cause. it is inherited in an autosomal dominant manner. treatment may include a pacemaker and implantable cardioverter defibrillator (icd) for arrhythmia or cardiac conduction defects; cardiac transplantation for progressive or life-threatening ...
Myopathy Treatment - Myopathies - HealthCommunities.comMyopathy Treatment - Myopathies - HealthCommunities.com
The treatment of myopathy depends on what causes the myopathy. Treatments range from drug therapy for the muscular dystrophies and inflammatory myopathies to simply avoiding situations that work the muscles too hard for the metabolic myopathies.
Calf numbness and Weak and soft muscles - Symptom Checker - check medical symptoms at RightDiagnosisCalf numbness and Weak and soft muscles - Symptom Checker - check medical symptoms at RightDiagnosis
List of 37 causes for Calf numbness and Weak and soft muscles, alternative diagnoses, rare causes, misdiagnoses, patient stories, and much more.
Anatomy | Key To Good HealthAnatomy | Key To Good Health
The muscular system can be broken down into three types of muscles: skeletal, smooth and cardiac. Skeletal muscles are the only voluntary muscle tissue in the human body and control every action that a person consciously performs. As the muscular system impacts so many of the functions necessary to sustain life, any muscle disorder can cause health problems, ranging from minor to severe. Not only do muscular disorders affect mobility, but can result in many other functional abnormalities, such as the inability to breath, swallow or speak. Causes of muscle disorders include injury or overuse, such as sprains or strains, cramps or tendinitis, a genetic disorder, such as muscular dystrophy, cancers, inflammation, such as myositis, diseases of nerves that affect muscles, infections, and certain medicines. Calf muscle - The calf muscle, on the back of the lower leg, is actually made up of two muscles: the gastrocnemius and the soleus. The gastrocnemius is the larger calf muscle, forming the bulge ...
Most recent papers with the keyword advance directive issues | Read by QxMDMost recent papers with the keyword advance directive issues | Read by QxMD
To reach the healthcare market and have a medical intervention reimbursed in any format carries high risk and very low success rates. Even when all regulatory hurdles have been surpassed, there is no guarantee that the product will be purchased; a different body makes that decision using criteria typically unknown to early-stage innovators and intervention developers. In the context of skeletal muscle diseases, the field is at a crossroads; accurate diagnoses are difficult to obtain, patient management and monitoring are equally difficult, cures are evasive, and disease progression is not well enough understood in the human to identify clear targets (irrespective of whether the specific muscle disease is rare or frequent because the progression is slow and the tissue large ...
2d7p - Proteopedia, life in 3D2d7p - Proteopedia, life in 3D
FLNC_HUMAN] Defects in FLNC are the cause of myopathy myofibrillar type 5 (MFM5) [MIM:609524]. A neuromuscular disorder, usually with an adult onset, characterized by focal myofibrillar destruction and pathological cytoplasmic protein aggregations, and clinical features of a limb-girdle myopathy.[1] Defects in FLNC are the cause of myopathy distal type 4 (MPD4) [MIM:614065]. MPD4 is a slowly progressive muscular disorder characterized by distal muscle weakness and atrophy affecting the upper and lower limbs. Onset occurs around the third to fourth decades of life, and patients remain ambulatory even after long disease duration. Muscle biopsy shows non-specific changes with no evidence of rods, necrosis, or inflammation.[2] ...
Study Found L- carnitine Can Heal Injured Heart Muscles | Muscular Disorders discussions | Body & Health Conditions center |...Study Found L- carnitine Can Heal Injured Heart Muscles | Muscular Disorders discussions | Body & Health Conditions center |...
L-carnitine is a naturally occurring amino acid which is found naturally in red meats and according to a new study it could help in treating injured heart muscles.
muscle - Musculoskeletal vs. muscular disorders - Medical Sciences Stack Exchangemuscle - Musculoskeletal vs. muscular disorders - Medical Sciences Stack Exchange
Stack Exchange Inc., and its sites including Medical Sciences Stack Exchange, is not a medical practice or healthcare provider and does not provide medical advice. Stack Exchange Inc., and its sites including Medical Sciences Stack Exchange, does not endorse or recommend any healthcare providers that moderate or otherwise contribute to this Site. Consult your own doctor for medical advice. The information and opinions shared here do not reflect the views of Stack Exchange Inc. and are not provided nor endorsed by Stack Exchange Inc. The content on this site is solely provided by individual community members who are not posting on behalf of Stack Exchange Inc.. Do not share personal medical information, medical history or any other specific details about a persons medical symptoms, condition etc (whether yours or someone you know) on this site or any Stack Exchange site. This is a public Site and all posts on this Site can be seen by anyone and may be shared freely with others.. Medical Sciences ...
Incidence and Management of Exertional Rhabdomyolysis | TheHorse.comIncidence and Management of Exertional Rhabdomyolysis | TheHorse.com
|P> |P>Stephanie Valberg, DVM, PhD, professor of large animal medicine and director of the University of Minnesotas Equine Center, tackled the broad subject of skeletal muscle disease related to exercise at the 2006 AAEP Convention. |P>This has
Section 3. Nerve and Muscle Disorders | Harrisons Principles of Internal Medicine, 18e | AccessPharmacy | McGraw-Hill MedicalSection 3. Nerve and Muscle Disorders | Harrison's Principles of Internal Medicine, 18e | AccessPharmacy | McGraw-Hill Medical
Section 3. Nerve and Muscle Disorders. In: Longo DL, Fauci AS, Kasper DL, Hauser SL, Jameson J, Loscalzo J. Longo D.L., Fauci A.S., Kasper D.L., Hauser S.L., Jameson J, Loscalzo J Eds. Dan L. Longo, et al.eds. Harrisons Principles of Internal Medicine, 18e New York, NY: McGraw-Hill; 2012. http://accesspharmacy.mhmedical.com/content.aspx?bookid=331§ionid=40727200. Accessed February 19, 2018 ...
Muscle Pain and Weakness in Thyroid DiseaseMuscle Pain and Weakness in Thyroid Disease
Read about the link between muscle weakness and pain and thyroid disease, and how your muscle symptoms can be effectively treated.
Buy Skelaxin Online | Best Canadian PharmacyBuy Skelaxin Online | Best Canadian Pharmacy
Skelaxin belongs to the group of miorelaxants. Its main active substance blocks the nerve impulses (or pain feeling) in the brain, which contributes to the muscle relaxation. It is especially efficient when used together with physiotherapy and rest, to treat the severe skeletal muscle disorders. Muscle cramps may also be the reason of back pain. It provides fast relief from pain without any negative adverse effects.
Muscle-Related Statin Effects Seem to Correlate With Potency - MPRMuscle-Related Statin Effects Seem to Correlate With Potency - MPR
The rates of muscle-related adverse effects (AEs) differ greatly for various statins, with the highest reported rates seen for rosuvastatin.
Muscle DiseasesMuscle Diseases
There are a large number of congenital myopathies in the literature for which the genes have not yet been identified Table 1.2 Engel et al., 1970 Brooke and Neville, 1972 Engel et al., 1972 Lake and Wilson, 1975 Fardeau et al., 1976 Ringel et al., 1978 Carpenter et al., 1979 Fidzianska et al., 1981 Goebel et al., 1981 Mrak et al., 1993 Mrak et al., 1996 Marbini et al., 1998 Bourque et al., 1999 Goebel and Anderson, 1999 Ikezoe et al., 2000 Selcen et al., 2001 Gommans et al., 2003 . All of these.... ...
What Is Causing This Womans Severe Muscle Pain? - The New York TimesWhat Is Causing This Woman's Severe Muscle Pain? - The New York Times
Help Dr. Lisa Sanders get to the bottom of unsolved medical mysteries. This week: What is causing this womans severe muscle pain?
Muscle ConditionsMuscle Conditions
Alterations of potassium and magnesium availability in the body are decisive causes of the onset of muscle symptoms, loss of strength and cramps
CoQ10 for Cardiovascular Disease | ISCMRCoQ10 for Cardiovascular Disease | ISCMR
CoQ10 is believed to improve mitochondrial energy production as well as endothelial function. It has been used for many indications, including primary prevention of CVD, treatment of acute myocardial infarction (MI), muscle symptoms associated with statins, and in some types of cancers. New data are also emerging about CoQ10s effects in chronic heart failure. Read more here.. ...
Cholesterol Drug & Muscle Pain - Share The WealthCholesterol Drug & Muscle Pain - Share The Wealth
I have been on Crestor for quite a while. Within the past few months I developed dibilating muscle pain and stiffness. So much so that I literally "tripped over my own feet" because I couldnt lift my legs. I fell and hurt myself pretty badly. Frankly I thought I had a muscle disorder like ALS. The symptoms seemed to be the same. Although I had always heard the disclaimer about muscle pain on Crestor commercials but it was an article on AOL (last week) that saved me. I clicked on it and the article descrbed my symptoms, and how I felt, perfectly. In fact it said that these symptoms can mimic ALS and/or Muscular Dystrohpy. I work in a hospital so I ran down to the primary care clinic and told the doctor my symptoms and how I felt. Thank goodness he believed me. My blood tests came back negative, however I am in a small group that have the symptoms but normal blood tests. I havent taken Crestor for about a week now. I feel like a different person! In fact I started feeling better a couple of days ...
Managing lipidsManaging lipids
Statin treatment is very common in Australia. What is the role of absolute cardiovascular risk assessment and how can we address suspected statin-associated muscle symptoms?
Muscle soreness or more serious? | SaukValley.comMuscle soreness or more serious? | SaukValley.com
Last week I tried a new circuit-style workout that combined cardio with strength training. I had some muscle soreness for the next couple of days, but I knew it was a sign that my workout had been effective.
God And My Muscle Disease - Part 1 | The Listening PostGod And My Muscle Disease - Part 1 | The Listening Post
After hinting at or mentioning my muscle disease in quite a few of my articles, I think I should finally sit down and explain in more detail what exactly this disease is, how it affects me, and where I think God is in the midst of all this. As you read this story, please do…
myalgia - Postsmyalgia - Posts
myalgia - MedHelps myalgia Center for Information, Symptoms, Resources, Treatments and Tools for myalgia. Find myalgia information, treatments for myalgia and myalgia symptoms.
Systemic Lupus Erythematosus - Bone, Joint, and Muscle Disorders - Merck Manuals Consumer VersionSystemic Lupus Erythematosus - Bone, Joint, and Muscle Disorders - Merck Manuals Consumer Version
Learn about the causes, symptoms, diagnosis & treatment of Autoimmune Disorders of Connective Tissue from the Home Version of the Merck Manuals.
Bone, Joint, and Muscle Disorders in Horses - Veterinary ManualBone, Joint, and Muscle Disorders in Horses - Veterinary Manual
Merck and Co., Inc., Kenilworth, NJ, USA (known as MSD outside the US and Canada) is a global healthcare leader working to help the world be well. From developing new therapies that treat and prevent disease to helping people in need, we are committed to improving health and well-being around the world. The Manual was first published in 1899 as a service to the community. The legacy of this great resource continues as the Merck Manual in the US and Canada and the MSD Manual in the remainder of the world. Learn more about our commitment to Global Medical Knowledge.. ...
Infectious Arthritis - Bone, Joint, and Muscle Disorders - Merck Manuals Consumer VersionInfectious Arthritis - Bone, Joint, and Muscle Disorders - Merck Manuals Consumer Version
Learn about the causes, symptoms, diagnosis & treatment of Bone and Joint Infections from the Home Version of the Merck Manuals.
AnalgesicAnalgesic
Lactate dehydrogenaseLactate dehydrogenase
Retinitis pigmentosaRetinitis pigmentosa
Boris PetrikovskyBoris Petrikovsky
HardgainerHardgainer
Familial partial lipodystrophyFamilial partial lipodystrophy
LMNA - 维基百科，自由的百科全书LMNA - 维基百科，自由的百科全书
LMNALMNA
Acceleron PharmaAcceleron Pharma
Macrophagic myofasciitisMacrophagic myofasciitis
Hereditary motor and sensory neuropathyHereditary motor and sensory neuropathy
Horse colicHorse colic
Heart failureHeart failure
Christian Friedrich von KahlbutzChristian Friedrich von Kahlbutz
LDB3LDB3
Hereditary inclusion body myopathyHereditary inclusion body myopathy
CamptocormiaCamptocormia
TonicityTonicity
AtrophyAtrophy
MyostatinMyostatin
NeuromechanicsNeuromechanics
CathepsinCathepsin
Ludvig StrigeusLudvig Strigeus
Introduction to geneticsIntroduction to genetics
UBA1UBA1
FollistatinFollistatin
Orphan drugOrphan drug
NusinersenNusinersen
WHDWHD
Ronald SukenickRonald Sukenick
AnatomyDiseasesChemicals and DrugsPsychiatry and PsychologyPhenomena and ProcessesDisciplines and OccupationsAnthropology, Education, Sociology and Social PhenomenaHumanitiesInformation ScienceHealth CareGeographicals
Muscular DiseasesMovementPostureHuman BodyMusclesSubstance PMuscle, SkeletalMuscle ProteinsNeuromuscular DiseasesMuscular DystrophiesMuscular Dystrophy, DuchenneHospitals, VoluntaryMuscular Dystrophy, AnimalHospitals, ProprietaryMyotonic DystrophyMuscular Dystrophy, FacioscapulohumeralChromosomes, Human, Pair 4DystrophinMuscular Atrophy, SpinalSpinal Muscular Atrophies of ChildhoodSurvival of Motor Neuron 1 ProteinSMN Complex ProteinsSurvival of Motor Neuron 2 ProteinHuntington DiseaseAtrophyLos AngelesAmerican Heart AssociationCardiologyCongresses as TopicHeartMotor NeuronsMotor Neuron DiseaseSarcoglycansMuscular Dystrophies, Limb-GirdleCalpainDystroglycansBiological Ontologies
Mahamasha thailam-Ayurvedic Oil for Neurological & Muscular Diseases | Ayurvedic ProductsMahamasha thailam-Ayurvedic Oil for Neurological & Muscular Diseases | Ayurvedic Products
Muscular System: Facts, Functions & DiseasesMuscular System: Facts, Functions & Diseases
Search of: Muscular Diseases - List Results - ClinicalTrials.govSearch of: 'Muscular Diseases' - List Results - ClinicalTrials.gov
The nuclear envelope in muscular dystrophy and cardiovascular diseases. - PubMed - NCBIThe nuclear envelope in muscular dystrophy and cardiovascular diseases. - PubMed - NCBI
ASU Researchers Discover New Path To Address Genetic Muscular Diseases - RedorbitASU Researchers Discover New Path To Address Genetic Muscular Diseases - Redorbit
List of Muscular System Diseases | LIVESTRONG.COMList of Muscular System Diseases | LIVESTRONG.COM
Types of Muscular Dystrophy and Neuromuscular Diseases | Johns Hopkins MedicineTypes of Muscular Dystrophy and Neuromuscular Diseases | Johns Hopkins Medicine
muscular skeletal chest pains - Heart Disease - MedHelpmuscular skeletal chest pains - Heart Disease - MedHelp
About Neuromuscular Diseases | Muscular Dystrophy AssociationAbout Neuromuscular Diseases | Muscular Dystrophy Association
Muscular-skeletal diseases | Health and medicine | Science | Khan AcademyMuscular-skeletal diseases | Health and medicine | Science | Khan Academy
Common Diseases of the Muscular & Skeletal System | Livestrong.comCommon Diseases of the Muscular & Skeletal System | Livestrong.com
Journal News: JBC: Cow muscular diseaseJournal News: JBC: Cow muscular disease
Types of Muscular Dystrophy and Neuromuscular DiseasesTypes of Muscular Dystrophy and Neuromuscular Diseases
Disease-specific and glucocorticoid-responsive serum biomarkers for Duchenne Muscular Dystrophy | Scientific ReportsDisease-specific and glucocorticoid-responsive serum biomarkers for Duchenne Muscular Dystrophy | Scientific Reports
How Effective is Stem Cell Therapy For Muscular Dystrophy - DiseasesHow Effective is Stem Cell Therapy For Muscular Dystrophy - Diseases
facioscapulohumeral muscular dystrophy Disease Ontology Browser - DOID:11727facioscapulohumeral muscular dystrophy Disease Ontology Browser - DOID:11727
Rare Neurological Diseases: Spinal Muscular Atrophy and Huntingtons DiseaseRare Neurological Diseases: Spinal Muscular Atrophy and Huntington's Disease
Study describes new muscular disease caused by mutation in myoglobin geneStudy describes new muscular disease caused by mutation in myoglobin gene
Conditions and Diseases -- Skeletal and Muscular Systems | St. Davids HealthCareConditions and Diseases -- Skeletal and Muscular Systems | St. David's HealthCare
10: Spinal Muscular Atrophy (SMA) - Top 10 Rare Diseases | HowStuffWorks10: Spinal Muscular Atrophy (SMA) - Top 10 Rare Diseases | HowStuffWorks
Privacy Policy - Muscular Diseases AwarenessPrivacy Policy - Muscular Diseases Awareness
Is Spinal Muscular Atrophy a Disease of Sensory Neurons? | ALZFORUMIs Spinal Muscular Atrophy a Disease of Sensory Neurons? | ALZFORUM
Double-deficient mice clarify Duchenne muscular dystrophy DMD disease processDouble-deficient mice clarify Duchenne muscular dystrophy DMD disease process
Translational Study Demonstrates Value of MSOT Imaging for the Assessment of Disease Progression in Duchenne Muscular DystrophyTranslational Study Demonstrates Value of MSOT Imaging for the Assessment of Disease Progression in Duchenne Muscular Dystrophy
Petition · We need Spinraza in pakistan, A treatment for rare disease Spinal Muscular Atrophy · Change.orgPetition · We need Spinraza in pakistan, A treatment for rare disease Spinal Muscular Atrophy · Change.org
Petition · Make rare disease treatment SPINRAZA,for SPINAL MUSCULAR ATROPHY available in SOUTH AFRICA · Change.orgPetition · Make rare disease treatment SPINRAZA,for SPINAL MUSCULAR ATROPHY available in SOUTH AFRICA · Change.org
Muscular dystrophy could be the next disease to get whacked by gene therapy - MIT Technology ReviewMuscular dystrophy could be the next disease to get whacked by gene therapy - MIT Technology Review
Study Shows Promise in Treating Heart Disease Associated With Muscular Dystrophy - NBC Los AngelesStudy Shows Promise in Treating Heart Disease Associated With Muscular Dystrophy - NBC Los Angeles
Kennedys Disease (Bulbospinal Muscular Atrophy, X-Linked Spinal and Bulbar Muscular Atrophy)Kennedy's Disease (Bulbospinal Muscular Atrophy, X-Linked Spinal and Bulbar Muscular Atrophy)
JCI - Muscle-specific SMN reduction reveals motor neuron-independent disease in spinal muscular atrophy modelsJCI - Muscle-specific SMN reduction reveals motor neuron-independent disease in spinal muscular atrophy models
autosomal recessive limb-girdle muscular dystrophy type 2M Disease Ontology Browser - DOID:0110296autosomal recessive limb-girdle muscular dystrophy type 2M Disease Ontology Browser - DOID:0110296
AnatomyDiseasesChemicals and DrugsPsychiatry and PsychologyPhenomena and ProcessesDisciplines and OccupationsAnthropology, Education, Sociology and Social PhenomenaHumanitiesInformation ScienceHealth CareGeographicals
Muscular DiseasesMovementPostureHuman BodyMusclesSubstance PMuscle, SkeletalMuscle ProteinsNeuromuscular DiseasesMuscular DystrophiesMuscular Dystrophy, DuchenneHospitals, VoluntaryMuscular Dystrophy, AnimalHospitals, ProprietaryMyotonic DystrophyMuscular Dystrophy, FacioscapulohumeralChromosomes, Human, Pair 4DystrophinMuscular Atrophy, SpinalSpinal Muscular Atrophies of ChildhoodSurvival of Motor Neuron 1 ProteinSMN Complex ProteinsSurvival of Motor Neuron 2 ProteinHuntington DiseaseAtrophyLos AngelesAmerican Heart AssociationCardiologyCongresses as TopicHeartMotor NeuronsMotor Neuron DiseaseSarcoglycansMuscular Dystrophies, Limb-GirdleCalpainDystroglycansBiological Ontologies
Spinal musculaDuchenne MuscularFacioscapulohumeral MuscularDystrophy AssociationAffected with muscular dystrophyDystrophiesParent Project Muscular DystrophyCaused by muscular dystrophyCongenital muscularForm of muscularTypes of muscularMyotonicMitochondrialNeurologicalProgressionTherapiesGenetic disease calledCardiovascularLymphomaOrphanNeurodegenerative diseasesPatientAlzheimer'sNeuromuscular disease researchRespiratoryTreatmentHumansGeneDifferent diseasesParkinson's DiseaseDegeneration2016OccursMusclesMultipleTreatmentsPatientsDystrophinRange of diseasesAffectsMuscle diseasesRare
Spinal muscula6
Duchenne Muscular32
Facioscapulohumeral Muscular3
Dystrophy Association4
Affected with muscular dystrophy1
Dystrophies4
Parent Project Muscular Dystrophy2
Caused by muscular dystrophy1
Congenital muscular1
  • At the American Society of Human Genetics conference, I will be presenting our work on a mutation-independent strategy to upregulate expression of a compensatory disease-modifying gene in Congenital Muscular Dystrophy type 1A (MDC1A) using a CRISPR/dCas9-based transcriptional activation system. (mousephenotype.org)
Form of muscular3
Types of muscular3
Myotonic2
Mitochondrial2
Neurological2
Progression6
  • Although the disease course may be variable, FSHD is most typically characterized by relatively slow disease progression. (hekimce.com)
  • There have been small studies that show that in different cases, nonstrenuous exercise may improve things like quality of life, but there's never been anything to show that it slows disease progression," says Elman. (everydayhealth.com)
  • In this work, we also demonstrate that dystrophic features and disease progression were significantly improved and partially reversed when the treatment was initiated in symptomatic 3-week old dy2j/dy2j mice with already-apparent hind limb paralysis and significant muscle fibrosis. (mousephenotype.org)
  • Findings from this study suggest that the [11C]PBR38 ligand, in particular, may be useful in detecting progression from mild cognitive impairment or treatment response in Alzheimer's Disease. (fnih.org)
  • treatment targets your child's specific symptoms to slow disease progression and improve quality of life. (dukehealth.org)
  • Diagnosed with Huntington's chorea more than six years ago, Althea has the desire and drive to work with Interim HealthCare's physical therapist and aides to help maintain her motor skills and slowing the progression of her disease. (interimhealthcare.com)
Therapies5
Genetic disease called1
Cardiovascular1
Lymphoma1
  • Non-Hodgkin's lymphoma, Hodgkins disease, Lyme disease, and muscular dystrophy warriors, survivors, patients, caregivers, and supporters, stand strong in the fight for a cure with our Fight Like a Girl Signature t-shirt in lime green with lime green awareness ribbon and boxing glove. (morgellonssurvey.org)
Orphan3
  • The company sought approval for deflazacort as an "orphan drug," a special approval pathway intended to encourage the development of drugs for very rare diseases. (washingtonpost.com)
  • This also seems to be another example of gaming the Orphan Drug Act, which was intended to try and encourage research into new therapeutic entities for people who have rare diseases - and it doesn't seem like this is that. (washingtonpost.com)
  • For example, tetrabenazine, a drug that was available from abroad and used for years to treat the uncontrollable tremors of Huntington's disease, was approved as an orphan drug in 2008. (washingtonpost.com)
Neurodegenerative diseases3
  • Over the past decade, the role of TDP-43 dysregulation and aggregation in nerve cells in neurodegenerative diseases such as ALS has become increasingly clear. (mda.org)
  • Motor neurone disease shares multiple commonalities with other neurodegenerative diseases and is an excellent model for evaluating all forms of neuroprotective treatment. (sheffield.ac.uk)
  • The Sheffield Institute for Translational Neuroscience (SITraN) was awarded the Queen's Anniversary Prize 2019 for delivering real benefits in improving patient outcomes for people living with some of the most devastating neurodegenerative diseases. (sheffield.ac.uk)
Patient1
  • In April 2015, in preparation for the FDA's Gastrointestinal Disorders Patient-Focused Drug Development Workshop , the second-ever FDA meeting addressing celiac disease, CDF surveyed our community . (celiac.org)
Alzheimer's7
  • The Foundation for the National Institutes of Health (FNIH) Biomarkers Consortium's Plasma Abeta project will evaluate next generation plasma Aβ assays to determine whether plasma Aβ peptide ratios increase the probability of identifying patients with amyloid positivity to improve clinical trial screening efficiency and reduce clinical trial costs for early stages of Alzheimer's Disease. (fnih.org)
  • The National Institutes of Health (NIH) convened the 3rd Alzheimer's Disease-Related Dementia Summit, that was held on March 14-15, 2019 in Bethesda, Maryland. (fnih.org)
  • This complements current efforts in the areas of Alzheimer's disease, type 2 diabetes and the autoimmune disorders of rheumatoid arthritis and systemic lupus erythematosus (lupus) . (fnih.org)
  • The Accelerating Medicines Partnership Alzheimer's Disease Project (AMP-AD) is a precompetitive partnership among government, industry, and nonprofit organizations that focuses on discovering novel, clinically relevant therapeutic targets and on developing biomarkers to help validate existing therapeutic targets. (fnih.org)
  • This project will aim to standardize and validate measurement methods for inflammatory markers associated with Alzheimer's Disease and/or Major Depressive Disorder to ultimately identify a unique biosignature of disease. (fnih.org)
  • The Biomarkers Consortium's Longitudinal CSF Proteomics Project, completed in Q4Y20, was the third stage of a multi-phased effort utilizing samples from the Alzheimer's Disease Neuroimaging Initiative (ADNI) expanding on the identification of promising proteins in a previous Biomarkers Consortium project to provide early validation for candidate AD biomarkers. (fnih.org)
  • Specifically, a married 30-year-old geneticist who will almost certainly lose her mind to early-onset Alzheimer's disease by age 40 chose to have her embryos tested in vitro for the disease gene. (readthehook.com)
Neuromuscular disease research1
  • This round of grant funding reinforces MDA's unwavering commitment to the progress of neuromuscular disease research and builds on the more than $1 billion MDA has already invested in research to uncover new treatments and cures for NMDs since its inception. (mda.org)
Respiratory1
Treatment3
  • Modified messenger RNA (mRNA) for expressing clinically beneficial proteins is an emerging new therapeutic approach that enables the body to produce therapeutic protein, opening up new treatment options for a wide range of diseases. (artbioscience.com)
  • Collectively, our data demonstrate the feasibility and therapeutic benefit of CRISPR/dCas9-mediated modulation of a disease modifier gene, which opens up an entirely new and mutation-independent treatment approach for all MDC1A patients. (mousephenotype.org)
  • An old steroid treatment, long available outside the United States, received approval this week for a rare disease that afflicts about 15,000 Americans. (washingtonpost.com)
Humans1
  • It's the first time the procedure was successful with an animal as large as a dog - an important step towards possible use on humans affected by the disease. (cbc.ca)
Gene5
  • Our teams have key skills in cellular and molecular biology, in vitro and in vivo disease modelling, human neuropathology, viral vector technology, genetics, pharmacology, gene therapy, RNA processing, electrophysiology, together with experience in clinical research and conducting clinical trials. (sheffield.ac.uk)
  • Huntington's disease is caused by carrying a faulty version of the gene for the Huntingtin protein, but the messenger. (thenakedscientists.com)
  • The idea behind this work is to identify the key molecular switches that can cause a gene to produce, or not produce, a protein-and, ideally, use that knowledge to treat a disease. (xconomy.com)
  • The Bespoke Gene Therapy Consortium (BGTC) is a developing public-private partnership dedicated to making gene therapy a reality for people with rare genetic diseases affecting populations too small to be viable from the current commercial perspective. (fnih.org)
  • The mother in this case certainly knows what would face any child of hers born with the disease gene. (readthehook.com)
Different diseases1
  • Can the fledgling company surpass previous efforts and develop epigenetic drugs for a slew of different diseases? (xconomy.com)
Parkinson's Disease4
Degeneration2
20161
  • This is an emerging field that's just starting to realize its full potential of treating human disease," Fulcrum's CEO Robert Gould, who once ran Epizyme, told Xconomy in 2016 . (xconomy.com)
Occurs1
Muscles2
Multiple1
Treatments2
  • The biotech sector is projected to surge beyond $775 billion by 2024 as scientists develop treatments for thousands of diseases. (yahoo.com)
  • The FDA Gastroenterology Regulatory Endpoints and Advancement of Therapeutics (GREAT3) Workshop served to formalize the discussion of therapeutic treatments for celiac disease. (celiac.org)
Patients5
Dystrophin1
Range of diseases1
  • The Neuroscience Institute gives us a new opportunity to build on the existing expertise and advances within SITraN and apply them to a wider community of academic excellence tackling a broader range of diseases. (sheffield.ac.uk)
Affects1
Muscle diseases2
Rare4
  • Mostly, Dr. Smith says, that's because Duchenne is a rare disease, and there isn't anything that can be done about a girl or woman's status as a carrier even if it's discovered. (everydayhealth.com)
  • In theory, these vouchers exist for a good reason: Regulators want to encourage companies to invest in developing drugs for rare diseases that afflict children. (washingtonpost.com)
  • In recent years, companies that have gotten old or existing drugs approved to treat rare diseases have reaped big financial rewards. (washingtonpost.com)
  • Ghias said that the company carefully thought about pricing and came up with a price far lower than other rare disease drugs - in the bottom 10th percentile of rare disease drug prices. (washingtonpost.com)
Microarray Analysis of Gene Expression by Skeletal Muscle of Three Mouse Models of Kennedy Disease/Spinal Bulbar Muscular...
Microarray Analysis of Gene Expression by Skeletal Muscle of Three Mouse Models of Kennedy Disease/Spinal Bulbar Muscular... (journals.plos.org)
Muscular System: Facts, Functions & Diseases
Muscular System: Facts, Functions & Diseases (livescience.com)
Anti-inflammatory activity of natural dietary flavonoids - Food & Function (RSC Publishing) DOI:10.1039/C0FO00103A
Anti-inflammatory activity of natural dietary flavonoids - Food & Function (RSC Publishing) DOI:10.1039/C0FO00103A (pubs.rsc.org)
Moderate Muscle Strength Key to Type 2 Diabetes Prevention
Moderate Muscle Strength Key to Type 2 Diabetes Prevention (medscape.com)
Experimental Models of Duchenne Muscular Dystrophy: Relationship with Cardiovascular Disease
Experimental Models of Duchenne Muscular Dystrophy: Relationship with Cardiovascular Disease (benthamopen.com)
November 2010 - Volume 16 - Issue 6 : The Neurologist
November 2010 - Volume 16 - Issue 6 : The Neurologist (journals.lww.com)
Pathology of Motor Neuron Disorders: Definition, Etiology, Epidemiology
Pathology of Motor Neuron Disorders: Definition, Etiology, Epidemiology (emedicine.medscape.com)
Disease-specific and glucocorticoid-responsive serum biomarkers for Duchenne Muscular Dystrophy | Scientific Reports
Disease-specific and glucocorticoid-responsive serum biomarkers for Duchenne Muscular Dystrophy | Scientific Reports (nature.com)
Dr Oliver Duke. Consultant Rheumatologist and General Physician. Specialist in joint, bone and muscular diseases.
Dr Oliver Duke. Consultant Rheumatologist and General Physician. Specialist in joint, bone and muscular diseases. (painfuljointdoc.com)
William L. Oppenheim, MD : Pediatric Orthopaedic Surgery - Santa Monica, California
William L. Oppenheim, MD : Pediatric Orthopaedic Surgery - Santa Monica, California (uclahealth.org)
Compound Report Card
Compound Report Card (ebi.ac.uk)
Analgesic - Wikipedia
Analgesic - Wikipedia (en.wikipedia.org)
Tadalafil (Cialis) News, Research
Tadalafil (Cialis) News, Research (news-medical.net)
Epigenetics of neurodegenerative diseases - Wikipedia
Epigenetics of neurodegenerative diseases - Wikipedia (en.wikipedia.org)
Salk Scientists Redesign CRISPR to Create Precision Medicine Tool [image] | EurekAlert! Science News
Salk Scientists Redesign CRISPR to Create Precision Medicine Tool [image] | EurekAlert! Science News (eurekalert.org)
Genetic Disorder News, Research
Genetic Disorder News, Research (news-medical.net)
Muscular Dystrophy Signs and Symptoms | Everyday Health
Muscular Dystrophy Signs and Symptoms | Everyday Health (everydayhealth.com)
Muscle Disorders | MedlinePlus
Muscle Disorders | MedlinePlus (medlineplus.gov)
Fibromyalgia Archives - Redorbit
Fibromyalgia Archives - Redorbit (redorbit.com)
Adolescent Fibromyalgia Study - Full Text View - ClinicalTrials.gov
Adolescent Fibromyalgia Study - Full Text View - ClinicalTrials.gov (clinicaltrials.gov)
Video Gallery : Plastic and Reconstructive Surgery
Video Gallery : Plastic and Reconstructive Surgery (journals.lww.com)
Video Gallery : Plastic and Reconstructive Surgery
Video Gallery : Plastic and Reconstructive Surgery (journals.lww.com)