Intestine, Small
Intestines
Intestine, Large
Intestinal Mucosa
Jejunum
Ileum
Duodenum
Colon
Enterocytes
Digestive System
Cecum
Liver
Swine
Tissue Distribution
Stomach
Serous Membrane
Gastrointestinal Tract
RNA, Messenger
Caco-2 Cells
Organ Specificity
Paneth Cells
Intestinal Secretions
Epithelium
Molecular Sequence Data
Animals, Suckling
Myenteric Plexus
Feces
Digestive System Physiological Phenomena
Gastrointestinal Transit
Enteric Nervous System
Rats, Inbred Strains
Biological Transport
Mesentery
Rats, Sprague-Dawley
Gastrointestinal Hormones
Epithelial Cells
Immunohistochemistry
Rats, Wistar
Base Sequence
Kidney
Amino Acid Sequence
Peristalsis
Enteroendocrine Cells
Mice, Knockout
Diarrhea
Submucous Plexus
Sodium-Glucose Transporter 1
Muscle, Smooth
Glucose Transporter Type 5
Mucins
Bile Acids and Salts
Gene Expression Regulation
Myoelectric Complex, Migrating
Rabbits
Bile
Goblet Cells
Immunity, Mucosal
Guinea Pigs
Trichinellosis
Intestinal Polyps
Disease Models, Animal
Carrier Proteins
Sucrase-Isomaltase Complex
Cloning, Molecular
Chickens
Reverse Transcriptase Polymerase Chain Reaction
Interstitial Cells of Cajal
Symporters
Sodium
Hydrogen-Ion Concentration
Cholecystokinin
Absorption
Intestinal Obstruction
Gene Expression
Biological Transport, Active
Glucose
Mucin-2
Pancreas
Starch
Enterocolitis, Necrotizing
Mice, Transgenic
Lactase
Lactase-Phlorizin Hydrolase
Glucagon-Like Peptide 2
Celiac Disease
Intubation, Gastrointestinal
Cattle
Colitis
DNA, Complementary
Body Weight
Alkaline Phosphatase
Fatty Acids, Volatile
Mice, Inbred Strains
Weaning
Biological Availability
Species Specificity
Microsomes
Natriuretic Peptides
Dogs
Immunoglobulin A
Fetus
Mucus
Crohn Disease
In Situ Hybridization
Bacteria
Microscopy, Electron
Probiotics
Escherichia coli
Dietary Fiber
Enterocolitis
Intestinal Diseases, Parasitic
Homeostasis
Amino Acids
Glucagon-Like Peptides
Blotting, Northern
Enterotoxins
Glucuronides
Dose-Response Relationship, Drug
Bacterial Adhesion
Lymphoid Tissue
Metagenome
Ileostomy
Random Allocation
Boidae
S100 Calcium Binding Protein G
Chromatography, High Pressure Liquid
Inflammatory Bowel Diseases
Permeability
Sodium-Phosphate Cotransporter Proteins, Type IIb
Lactobacillus
Blotting, Western
Reperfusion Injury
Bifidobacterium
Bacterial Translocation
Fructose
Sequence Homology, Amino Acid
Genes, APC
Gene Expression Regulation, Developmental
Proglucagon
Polymerase Chain Reaction
Cell Differentiation
Colony Count, Microbial
Helminthiasis, Animal
Cholera Toxin
Receptors, Guanylate Cyclase-Coupled
Enterohepatic Circulation
Sheep
Fermentation
Aging
Histocytochemistry
Malabsorption Syndromes
Ulcer
Dietary Fats
Short Bowel Syndrome
Milk
Colostrum
Fluorescent Antibody Technique
Models, Biological
Cholesterol
Apolipoprotein B-48
Blood thymidine level and iododeoxyuridine incorporation and reutilization in DNA in mice given long-acting thymidine pellets. (1/10242)
A long-acting thymidine pellet consisting of 190 mg of cholesterol and 60 mg of thymidine has been developed for the study of thymidine metabolism and reutilization in vivo. Implantation of such a pellet s.c. in adult mice will maintain the blood plasma concentration of thymidine at levels between 40 and 8 X 10(-6) M, which are from 36 to 7 times those of normal mice, for periods up to 48 hr. During this period, in vivo uptake and reutilization of [125I]iododeoxyuridine, a thymidine analog, into intestinal and tumor DNA were almost completely suppressed. While iododeoxyuridine reutilization is not large in normal proliferative tissue even in the absence of pellet implants, reutilization of over 30% was measured in large, rapidly growing ascites tumors. The inhibition of iododeoxyuridine incorporation by elevated thymidine blood levels is directly proportional to serum concentration. This appears to be due to a thymidine pool in rapid equilibrium with blood thymidine. This pool is at least 10 times larger than the 4-nmole pool of extracellular thymidine. (+info)Transformation of intestinal epithelial cells by chronic TGF-beta1 treatment results in downregulation of the type II TGF-beta receptor and induction of cyclooxygenase-2. (2/10242)
The precise role of TGF-beta in colorectal carcinogenesis is not clear. The purpose of this study was to determine the phenotypic alterations caused by chronic exposure to TGF-beta in non-transformed intestinal epithelial (RIE-1) cells. Growth of RIE-1 cells was inhibited by >75% following TGF-beta1 treatment for 7 days, after which the cells resumed a normal growth despite the presence of TGF-beta1. These 'TGF-beta-resistant' cells (RIE-Tr) were continuously exposed to TGF-beta for >50 days. Unlike the parental RIE cells, RIE-Tr cells lost contact inhibition, formed foci in culture, grew in soft agarose. RIE-Tr cells demonstrated TGF-beta-dependent invasive potential in an in vitro assay and were resistant to Matrigel and Na-butyrate-induced apoptosis. The RIE-Tr cells were also tumorigenic in nude mice. The transformed phenotype of RIE-Tr cells was associated with a 95% decrease in the level of the type II TGF-beta receptor (TbetaRII) protein, a 40-fold increase in cyclooxygenase-2 (COX-2) protein, and 5.9-fold increase in the production of prostacyclin. Most RIE-Tr subclones that expressed low levels of TbetaRII and high levels of COX-2 were tumorigenic. Those subclones that express abundant TbetaRII and low levels of COX-2 were not tumorigenic in nude mice. A selective COX-2 inhibitor inhibited RIE-Tr cell growth in culture and tumor growth in nude mice. The reduced expression of TbetaRII, increased expression of COX-2, and the ability to form colonies in Matrigel were all reversible upon withdrawal of exogenous TGF-beta1 for the RIE-Tr cells. (+info)Vibrio parahaemolyticus thermostable direct hemolysin modulates cytoskeletal organization and calcium homeostasis in intestinal cultured cells. (3/10242)
Vibrio parahaemolyticus is a marine bacterium known to be the leading cause of seafood gastroenteritis worldwide. A 46-kDa homodimer protein secreted by this microorganism, the thermostable direct hemolysin (TDH), is considered a major virulence factor involved in bacterial pathogenesis since a high percentage of strains of clinical origin are positive for TDH production. TDH is a pore-forming toxin, and its most extensively studied effect is the ability to cause hemolysis of erythrocytes from different mammalian species. Moreover, TDH induces in a variety of cells cytotoxic effects consisting mainly of cell degeneration which often leads to loss of viability. In this work, we examined the cellular changes induced by TDH in monolayers of IEC-6 cells (derived from the rat crypt small intestine), which represent a useful cell model for studying toxins from enteric bacteria. In experimental conditions allowing cell survival, TDH induces a rapid transient increase in intracellular calcium as well as a significant though reversible decreased rate of progression through the cell cycle. The morphological changes seem to be dependent on the organization of the microtubular network, which appears to be the preferential cytoskeletal element involved in the cellular response to the toxin. (+info)Accumulation of astaxanthin all-E, 9Z and 13Z geometrical isomers and 3 and 3' RS optical isomers in rainbow trout (Oncorhynchus mykiss) is selective. (4/10242)
Concentrations of all-E-, 9Z- and 13Z- geometrical and (3R,3'R), (3R, 3'S) and (3S,3'S) optical isomers of astaxanthin were determined in rainbow trout liver, gut tissues, kidney, skin and blood plasma to evaluate their body distribution. Two cold-pelleted diets containing predominantly all-E-astaxanthin (36.9 mg/kg astaxanthin, 97% all-E-, 0.4% 9Z-, 1.5% 13Z-astaxanthin, and 1.1% other isomers, respectively) or a mixture of all-E- and Z-astaxanthins (35.4 mg/kg astaxanthin, 64% all-E-, 18.7% 9Z-, 12.3% 13Z-astaxanthin, and 2.0% other isomers, respectively), were fed to duplicate groups of trout for 69 d. Individual E/Z isomers were identified by VIS- and 1H-NMR-spectrometry, and quantified by high-performance liquid chromatography. Significantly higher total carotenoid concentration was observed in plasma of trout fed diets with all-E-astaxanthin (P < 0.05). The relative E/Z-isomer concentrations of plasma, skin and kidney were not significantly different among groups, whereas all-E-astaxanthin was higher in intestinal tissues and 13Z-astaxanthin was lower in liver of trout fed all-E-astaxanthin (P < 0.05). The relative amount of hepatic 13Z-astaxanthin (39-49% of total astaxanthin) was higher than in all other samples (P < 0.05). Synthetic, optically inactive astaxanthin was used in all experiments, and the determined dietary ratio between the 3R,3'R:3R, 3'S (meso):3S,3'S optical isomers was 25.3:49.6:25.1. The distribution of R/S-astaxanthin isomers in feces, blood, liver and fillet was similar to that in the diets. The ratio between (3S,3'S)- and (3R,3'R)-astaxanthin in the skin and posterior kidney was ca. 2:1 and 3:1, respectively, regardless of dietary E/Z-astaxanthin composition. The results show that geometrical and optical isomers of astaxanthin are distributed selectively in different tissues of rainbow trout. (+info)Energy depletion differently affects membrane transport and intracellular metabolism of riboflavin taken up by isolated rat enterocytes. (5/10242)
Isolated rat enterocytes, both normal and those de-energized with rotenone, were used to study the energy dependence of membrane and intracellular intestinal riboflavin transport in vitro. Membrane and intracellular transport were investigated by using short (3 min) and long (20 min) incubation times, respectively. For both types of cells and incubation times, [3H]-riboflavin uptake presented a saturable component prevailing at physiologic intraluminal concentrations. At 3 min incubation, saturable [3H]-riboflavin transport was apparently an energy-independent process with high affinity and low capacity. Values of the saturable component and its apparent constants, Km and Jmax, did not differ in normal and de-energized enterocytes. At 20 min incubation, saturable [3H]-riboflavin transport was a strictly energy-dependent process in which values of the saturable component were significantly greater in normal than in de-energized enterocytes. Km values did not differ in the two types of cells and were unmodified over 3 min, whereas in normal enterocytes, Jmax at 20 min [6.25 +/- 0.2 pmol/(mg protein. 20 min)] was significantly greater than at 3 min [2.67 +/- 0.33 pmol/(mg protein. 3 min)] and compared with de-energized enterocytes at 20 min [2.54 +/- 0.16 pmol/(mg protein. 20 min)]. Both membrane and intracellular events were inhibited by unlabeled riboflavin and analogs, which are good substrates for flavokinase, thus demonstrating the paramount role of this enzyme in riboflavin intestinal transport. (+info)The sodium concentration of enteral diets does not influence absorption of nutrients but induces intestinal secretion of water in miniature pigs. (6/10242)
Contradictory opinions exist as to whether the sodium concentration of enteral diets influences absorption of macronutrients and transepithelial movement of sodium and water. Therefore, we investigated the effects of various sodium concentrations of enteral diets on absorption of macronutrients and on net fluxes of sodium and water. In unanesthetized miniature pigs, a 150-cm jejunal segment was perfused with an oligopeptide (Peptisorb), an oligomeric and a polymeric diet. The polymeric diet was supplemented with pancreatic enzymes. The sodium concentrations varied between 30 and 150 mmol/L. The energy density was 3.4 MJ/L. The sodium concentration of the diets did not influence absorption of macronutrients and of total energy. However, increasing sodium concentrations of the diets were associated with increasing osmolality of the solutions, resulting in a linear increase in net secretion of water and flow rate of chyme. With all diets and sodium concentrations net secretion of sodium occurred. The sodium secretion was independent of the initial sodium concentration of the diets. It was linearly correlated with net flux of water and was largest in miniature pigs infused with the oligomeric diet. The sodium concentration of the jejunal effluent did not correspond to the initial sodium concentration of the diets. The present results indicate that enteral feeding of diets with high energy density inevitably increases net secretion of water and sodium as sodium concentration increases. Therefore, the sodium concentration of diets should be as low as possible to meet only the minimal daily requirement of sodium. Low sodium concentrations of diets have no negative effects on absorption of macronutrients. (+info)Molecular basis for the enterocyte tropism exhibited by Salmonella typhimurium type 1 fimbriae. (7/10242)
Salmonella typhimurium exhibits a distinct tropism for mouse enterocytes that is linked to their expression of type 1 fimbriae. The distinct binding traits of Salmonella type 1 fimbriae is also reflected in their binding to selected mannosylated proteins and in their ability to promote secondary bacterial aggregation on enterocyte surfaces. The determinant of binding in Salmonella type 1 fimbriae is a 35-kDa structurally distinct fimbrial subunit, FimHS, because inactivation of fimHS abolished binding activity in the resulting mutant without any apparent effect on fimbrial expression. Surprisingly, when expressed in the absence of other fimbrial components and as a translational fusion protein with MalE, FimHS failed to demonstrate any specific binding tropism and bound equally to all cells and mannosylated proteins tested. To determine if the binding specificity of Salmonella type 1 fimbriae was determined by the fimbrial shaft that is intimately associated with FimHS, we replaced the amino-terminal half of FimHS with the corresponding sequence from Escherichia coli FimH (FimHE) that contains the receptor binding domain of FimHE. The resulting hybrid fimbriae bearing FimHES on a Salmonella fimbrial shaft exhibited binding traits that resembled that of Salmonella rather than E. coli fimbriae. Apparently, the quaternary constraints imposed by the fimbrial shaft on the adhesin determine the distinct binding traits of S. typhimurium type 1 fimbriae. (+info)Gallstones: an intestinal disease? (8/10242)
Current evidence suggests that impaired intestinal motility may facilitate gallstone formation by influencing biliary deoxycholate levels or by modulating interdigestive gall bladder motility (fig 2), although a primary intestinal defect in gallstone pathogenesis has not yet been demonstrated. In the cold war period, most interesting events, from a political point of view, occurred at the border between capitalist and communist systems, near the iron curtain. Similarly, the gall bladder and biliary tract can be viewed as the border between liver and intestinal tract, where many interesting things occur with profound impact on both systems. Combined efforts by researchers in the field of hepatology and gastrointestinal motility should brake down the Berlin wall of ignorance of one of the most common diseases in the Western world. (+info)Types of Intestinal Neoplasms:
1. Adenomas: These are benign tumors that grow on the inner lining of the intestine. They can become malignant over time if left untreated.
2. Carcinomas: These are malignant tumors that develop in the inner lining of the intestine. They can be subdivided into several types, including colon cancer and rectal cancer.
3. Lymphoma: This is a type of cancer that affects the immune system and can occur in the intestines.
4. Leiomyosarcomas: These are rare malignant tumors that develop in the smooth muscle layers of the intestine.
Causes and Risk Factors:
The exact cause of intestinal neoplasms is not known, but several factors can increase the risk of developing these growths. These include:
1. Age: The risk of developing intestinal neoplasms increases with age.
2. Family history: Having a family history of colon cancer or other intestinal neoplasms can increase the risk of developing these growths.
3. Inflammatory bowel disease: People with inflammatory bowel diseases, such as ulcerative colitis and Crohn's disease, are at higher risk of developing intestinal neoplasms.
4. Genetic mutations: Certain genetic mutations can increase the risk of developing intestinal neoplasms.
5. Diet and lifestyle factors: A diet high in fat and low in fiber, as well as lack of physical activity, may increase the risk of developing intestinal neoplasms.
Symptoms:
Intestinal neoplasms can cause a variety of symptoms, including:
1. Abdominal pain or discomfort
2. Changes in bowel habits, such as diarrhea or constipation
3. Blood in the stool
4. Weight loss
5. Fatigue
6. Loss of appetite
Diagnosis:
To diagnose intestinal neoplasms, a doctor may perform several tests, including:
1. Colonoscopy: A colonoscope is inserted through the rectum and into the colon to visualize the inside of the colon and detect any abnormal growths.
2. Biopsy: A small sample of tissue is removed from the colon and examined under a microscope for cancer cells.
3. Imaging tests: Such as X-rays, CT scans, or MRI scans to look for any abnormalities in the colon.
4. Blood tests: To check for certain substances in the blood that are associated with intestinal neoplasms.
Treatment:
The treatment of intestinal neoplasms depends on the type and location of the growth, as well as the stage of the cancer. Treatment options may include:
1. Surgery: To remove the tumor and any affected tissue.
2. Chemotherapy: To kill any remaining cancer cells with drugs.
3. Radiation therapy: To kill cancer cells with high-energy X-rays or other forms of radiation.
4. Targeted therapy: To use drugs that target specific molecules on cancer cells to kill them.
5. Immunotherapy: To use drugs that stimulate the immune system to fight cancer cells.
Prognosis:
The prognosis for intestinal neoplasms depends on several factors, including the type and stage of the cancer, the location of the growth, and the effectiveness of treatment. In general, early detection and treatment improve the prognosis, while later-stage cancers have a poorer prognosis.
Complications:
Intestinal neoplasms can cause several complications, including:
1. Obstruction: The tumor can block the normal flow of food through the intestine, leading to abdominal pain and other symptoms.
2. Bleeding: The tumor can cause bleeding in the intestine, which can lead to anemia and other complications.
3. Perforation: The tumor can create a hole in the wall of the intestine, leading to peritonitis (inflammation of the lining of the abdomen) and other complications.
4. Metastasis: The cancer cells can spread to other parts of the body, such as the liver or lungs, and cause further complications.
5. Malnutrition: The tumor can make it difficult for the body to absorb nutrients, leading to malnutrition and other health problems.
Prevention:
There is no sure way to prevent intestinal neoplasms, but there are several steps that may help reduce the risk of developing these types of cancer. These include:
1. Avoiding known risk factors: Avoiding known risk factors such as smoking, excessive alcohol consumption, and a diet high in processed meat can help reduce the risk of developing intestinal neoplasms.
2. Maintaining a healthy diet: Eating a balanced diet that is high in fruits, vegetables, and whole grains can help keep the intestines healthy and may reduce the risk of cancer.
3. Exercise regularly: Regular exercise can help maintain a healthy weight, improve digestion, and may reduce the risk of developing intestinal neoplasms.
4. Managing chronic conditions: Managing chronic conditions such as inflammatory bowel disease, diabetes, and obesity can help reduce the risk of developing intestinal neoplasms.
5. Screening tests: Regular screening tests such as colonoscopy, CT scan, or barium enema can help detect precancerous polyps or early-stage cancer, allowing for early treatment and prevention of advanced disease.
Early detection and diagnosis are crucial for effective treatment and survival rates for intestinal neoplasms. If you have any of the risk factors or symptoms mentioned above, it is essential to consult a doctor as soon as possible. A thorough examination and diagnostic tests can help determine the cause of your symptoms and recommend appropriate treatment.
1. Irritable Bowel Syndrome (IBS): A chronic condition characterized by abdominal pain, bloating, and changes in bowel habits.
2. Inflammatory Bowel Disease (IBD): A group of chronic conditions that cause inflammation in the digestive tract, including Crohn's disease and ulcerative colitis.
3. Diverticulosis: A condition in which small pouches form in the wall of the intestine, often causing abdominal pain and changes in bowel habits.
4. Intestinal obstruction: A blockage that prevents food, fluids, and gas from passing through the intestine, often causing abdominal pain, nausea, and vomiting.
5. Intestinal ischemia: A reduction in blood flow to the intestine, which can cause damage to the tissues and lead to life-threatening complications.
6. Intestinal cancer: Cancer that develops in the small intestine or large intestine, often causing symptoms such as abdominal pain, weight loss, and rectal bleeding.
7. Gastrointestinal infections: Infections caused by viruses, bacteria, or parasites that affect the gastrointestinal tract, often causing symptoms such as diarrhea, vomiting, and abdominal pain.
8. Intestinal motility disorders: Disorders that affect the movement of food through the intestine, often causing symptoms such as abdominal pain, bloating, and constipation.
9. Malabsorption: A condition in which the body is unable to properly absorb nutrients from food, often caused by conditions such as celiac disease or pancreatic insufficiency.
10. Intestinal pseudo-obstruction: A condition in which the intestine becomes narrowed or blocked, often causing symptoms such as abdominal pain, bloating, and constipation.
These are just a few examples of the many potential complications that can occur when the gastrointestinal system is not functioning properly. It is important to seek medical attention if you experience any persistent or severe symptoms in order to receive proper diagnosis and treatment.
Symptoms of enteritis may include diarrhea, abdominal pain, fever, nausea, vomiting, and weight loss. In severe cases, the condition can lead to dehydration, electrolyte imbalances, and even death if left untreated.
The diagnosis of enteritis is typically made through a combination of physical examination, medical history, and diagnostic tests such as endoscopy, imaging studies, and laboratory tests (e.g., blood tests, stool cultures). Treatment depends on the underlying cause of the condition and may include antibiotics, anti-inflammatory medications, and supportive care to manage symptoms.
Benign jejal neoplasms include:
1. Adenomas: These are benign tumors that grow on the lining of the jejunum and can become cancerous over time if left untreated.
2. Villous adenomas: These are benign tumors that grow on the villi, which are small projections on the lining of the small intestine that increase the surface area for nutrient absorption.
3. GISTs (gastrointestinal stromal tumors): These are rare benign tumors that can occur in the jejunum and other parts of the digestive system.
Malignant jejal neoplasms include:
1. Adenocarcinomas: These are cancerous tumors that grow on the lining of the jejunum and can invade surrounding tissues and organs.
2. Lymphoma: This is a type of cancer that affects the immune system and can occur in the jejunum.
3. Leiomyosarcomas: These are rare cancerous tumors that grow on the muscular walls of the jejunum.
Jejunal neoplasms can cause symptoms such as abdominal pain, diarrhea, nausea, and vomiting, depending on their location and size. They can also cause anemia, bleeding, and blockage of the intestine if they become large enough to obstruct the passage of food and fluids.
Jejunal neoplasms are diagnosed through a combination of endoscopy, imaging tests such as CT scans or MRI, and biopsy. Treatment options depend on the type and location of the neoplasm, and can include surgery, chemotherapy, and radiation therapy.
Benign ileal neoplasms include:
1. Adenomas: These are growths that are similar to colon polyps and can develop into colon cancer if left untreated.
2. Villous adenomas: These are benign tumors that grow on the villi, which are small projections that line the inside of the intestine.
3. Lipomas: These are slow-growing, non-cancerous growths that are made up of fat cells.
Malignant ileal neoplasms include:
1. Adenocarcinoma: This is the most common type of small intestine cancer and accounts for about 95% of all cases. It can occur in any part of the small intestine, but is more common in the duodenum (the first part of the small intestine).
2. Squamous cell carcinoma: This type of cancer occurs in the upper parts of the small intestine and is less common than adenocarcinoma.
3. Neuroendocrine tumors: These are rare tumors that occur in the hormone-producing cells of the small intestine and can produce excess hormones that can cause symptoms such as diarrhea, abdominal pain, and weight loss.
Ileal neoplasms can cause a variety of symptoms depending on their size, location, and type. These may include:
* Abdominal pain or discomfort
* Diarrhea or constipation
* Weight loss or loss of appetite
* Fatigue or weakness
* Nausea or vomiting
* Abnormal bleeding or discharge from the rectum
If you suspect that you may have an ileal neoplasm, it is important to seek medical attention as soon as possible. A healthcare professional can perform a series of tests and examinations to diagnose and determine the appropriate treatment for your condition. These may include:
1. Endoscopy: A flexible tube with a camera and light on the end is inserted through the mouth or rectum to visualize the inside of the small intestine and look for any abnormalities.
2. Imaging tests: Such as X-rays, CT scans, or MRI scans to visualize the small intestine and look for any tumors or other abnormalities.
3. Biopsy: A sample of tissue is removed from the small intestine and examined under a microscope to determine if there are any cancer cells present.
4. Blood tests: To check for certain substances in the blood that can indicate the presence of a neoplasm.
5. Genetic testing: To look for genetic changes that may indicate the presence of a neoplasm.
Treatment for ileal neoplasms depends on the type, size, location, and stage of the tumor, as well as the patient's overall health. Treatment options may include:
1. Surgery: To remove the tumor and any affected tissue.
2. Chemotherapy: To kill cancer cells with drugs.
3. Radiation therapy: To kill cancer cells with high-energy X-rays or other forms of radiation.
4. Targeted therapy: To use drugs or other substances to target specific molecules on the surface of cancer cells.
5. Supportive care: To manage symptoms and side effects, such as pain, nausea, and vomiting.
It's important for patients with ileal neoplasms to work closely with their healthcare team to determine the best course of treatment for their specific condition. With prompt and appropriate treatment, many people with ileal neoplasms can achieve long-term survival and a good quality of life.
There are several types of diarrhea, including:
1. Acute diarrhea: This type of diarrhea is short-term and usually resolves on its own within a few days. It can be caused by a viral or bacterial infection, food poisoning, or medication side effects.
2. Chronic diarrhea: This type of diarrhea persists for more than 4 weeks and can be caused by a variety of conditions, such as irritable bowel syndrome (IBS), inflammatory bowel disease (IBD), or celiac disease.
3. Diarrhea-predominant IBS: This type of diarrhea is characterized by frequent, loose stools and abdominal pain or discomfort. It can be caused by a variety of factors, including stress, hormonal changes, and certain foods.
4. Infectious diarrhea: This type of diarrhea is caused by a bacterial, viral, or parasitic infection and can be spread through contaminated food and water, close contact with an infected person, or by consuming contaminated food.
Symptoms of diarrhea may include:
* Frequent, loose, and watery stools
* Abdominal cramps and pain
* Bloating and gas
* Nausea and vomiting
* Fever and chills
* Headache
* Fatigue and weakness
Diagnosis of diarrhea is typically made through a physical examination, medical history, and laboratory tests to rule out other potential causes of the symptoms. Treatment for diarrhea depends on the underlying cause and may include antibiotics, anti-diarrheal medications, fluid replacement, and dietary changes. In severe cases, hospitalization may be necessary to monitor and treat any complications.
Prevention of diarrhea includes:
* Practicing good hygiene, such as washing hands frequently and thoroughly, especially after using the bathroom or before preparing food
* Avoiding close contact with people who are sick
* Properly storing and cooking food to prevent contamination
* Drinking safe water and avoiding contaminated water sources
* Avoiding raw or undercooked meat, poultry, and seafood
* Getting vaccinated against infections that can cause diarrhea
Complications of diarrhea can include:
* Dehydration: Diarrhea can lead to a loss of fluids and electrolytes, which can cause dehydration. Severe dehydration can be life-threatening and requires immediate medical attention.
* Electrolyte imbalance: Diarrhea can also cause an imbalance of electrolytes in the body, which can lead to serious complications.
* Inflammation of the intestines: Prolonged diarrhea can cause inflammation of the intestines, which can lead to abdominal pain and other complications.
* Infections: Diarrhea can be a symptom of an infection, such as a bacterial or viral infection. If left untreated, these infections can lead to serious complications.
* Malnutrition: Prolonged diarrhea can lead to malnutrition and weight loss, which can have long-term effects on health and development.
Treatment of diarrhea will depend on the underlying cause, but may include:
* Fluid replacement: Drinking plenty of fluids to prevent dehydration and replace lost electrolytes.
* Anti-diarrheal medications: Over-the-counter or prescription medications to slow down bowel movements and reduce diarrhea.
* Antibiotics: If the diarrhea is caused by a bacterial infection, antibiotics may be prescribed to treat the infection.
* Rest: Getting plenty of rest to allow the body to recover from the illness.
* Dietary changes: Avoiding certain foods or making dietary changes to help manage symptoms and prevent future episodes of diarrhea.
It is important to seek medical attention if you experience any of the following:
* Severe diarrhea that lasts for more than 3 days
* Diarrhea that is accompanied by fever, blood in the stool, or abdominal pain
* Diarrhea that is severe enough to cause dehydration or electrolyte imbalances
* Diarrhea that is not responding to treatment
Prevention of diarrhea includes:
* Good hand hygiene: Washing your hands frequently, especially after using the bathroom or before preparing food.
* Safe food handling: Cooking and storing food properly to prevent contamination.
* Avoiding close contact with people who are sick.
* Getting vaccinated against infections that can cause diarrhea, such as rotavirus.
Overall, while diarrhea can be uncomfortable and disruptive, it is usually a minor illness that can be treated at home with over-the-counter medications and plenty of fluids. However, if you experience severe or persistent diarrhea, it is important to seek medical attention to rule out any underlying conditions that may require more formal treatment.
The symptoms of trichinellosis can vary depending on the severity of the infection and the number of parasites consumed. Mild cases may not exhibit any symptoms at all, while more severe cases can cause a range of symptoms including:
* Abdominal pain
* Diarrhea
* Fever
* Headache
* Muscle pain
* Skin rash
* Swelling of the face and eyelids
In severe cases, trichinellosis can lead to complications such as inflammation of the heart, brain, and liver, and can be fatal if left untreated.
Trichinellosis is diagnosed through a combination of physical examination, medical history, and laboratory tests such as blood tests or biopsies. Treatment typically involves antiparasitic drugs to kill the parasite, and supportive care to manage symptoms.
Prevention of trichinellosis primarily involves proper food handling and cooking practices, such as cooking meat to an internal temperature of at least 165°F (74°C) to kill any Trichinella parasites that may be present. Avoiding the consumption of raw or undercooked meat, particularly from wild game animals, can also help prevent the infection.
There are several types of intestinal polyps, including:
1. Adenomatous polyps: These are the most common type of polyp and have the potential to become malignant (cancerous) over time if left untreated.
2. Hyperplastic polyps: These polyps are benign and do not have the potential to become cancerous.
3. Inflammatory polyps: These polyps are caused by inflammation in the intestines and are typically seen in conditions such as ulcerative colitis or Crohn's disease.
4. Villous adenomas: These polyps are benign growths that occur on the villi (small projections) of the intestinal lining. They have the potential to become malignant over time if left untreated.
Intestinal polyps can cause a variety of symptoms, including abdominal pain, rectal bleeding, and changes in bowel movements. However, many people with intestinal polyps do not experience any symptoms at all. Intestinal polyps are typically detected during a colonoscopy or other imaging tests.
If you have been diagnosed with an intestinal polyp, your doctor may recommend one of the following treatments:
1. Watchful waiting: If your polyp is small and not causing any symptoms, your doctor may recommend monitoring it closely with regular colonoscopies to see if it grows or changes over time.
2. Removal: Polyps can be removed during a colonoscopy using a technique called endoscopic mucosal resection (EMR) or by surgery.
3. Chemoprevention: In some cases, your doctor may recommend medications such as aspirin or sulindac to help reduce the risk of polyps growing back.
It's important to note that while intestinal polyps are generally not cancerous, they can sometimes become malignant over time if left untreated. Therefore, it is important to follow your doctor's recommendations for monitoring and treatment closely.
1) They share similarities with humans: Many animal species share similar biological and physiological characteristics with humans, making them useful for studying human diseases. For example, mice and rats are often used to study diseases such as diabetes, heart disease, and cancer because they have similar metabolic and cardiovascular systems to humans.
2) They can be genetically manipulated: Animal disease models can be genetically engineered to develop specific diseases or to model human genetic disorders. This allows researchers to study the progression of the disease and test potential treatments in a controlled environment.
3) They can be used to test drugs and therapies: Before new drugs or therapies are tested in humans, they are often first tested in animal models of disease. This allows researchers to assess the safety and efficacy of the treatment before moving on to human clinical trials.
4) They can provide insights into disease mechanisms: Studying disease models in animals can provide valuable insights into the underlying mechanisms of a particular disease. This information can then be used to develop new treatments or improve existing ones.
5) Reduces the need for human testing: Using animal disease models reduces the need for human testing, which can be time-consuming, expensive, and ethically challenging. However, it is important to note that animal models are not perfect substitutes for human subjects, and results obtained from animal studies may not always translate to humans.
6) They can be used to study infectious diseases: Animal disease models can be used to study infectious diseases such as HIV, TB, and malaria. These models allow researchers to understand how the disease is transmitted, how it progresses, and how it responds to treatment.
7) They can be used to study complex diseases: Animal disease models can be used to study complex diseases such as cancer, diabetes, and heart disease. These models allow researchers to understand the underlying mechanisms of the disease and test potential treatments.
8) They are cost-effective: Animal disease models are often less expensive than human clinical trials, making them a cost-effective way to conduct research.
9) They can be used to study drug delivery: Animal disease models can be used to study drug delivery and pharmacokinetics, which is important for developing new drugs and drug delivery systems.
10) They can be used to study aging: Animal disease models can be used to study the aging process and age-related diseases such as Alzheimer's and Parkinson's. This allows researchers to understand how aging contributes to disease and develop potential treatments.
Here are some examples of jejunal diseases:
1. Crohn's disease: This is a chronic inflammatory bowel disease that can affect any part of the gastrointestinal tract, including the jejunum. It causes inflammation and damage to the lining of the intestine, leading to symptoms such as diarrhea, abdominal pain, and fatigue.
2. Ulcerative colitis: This is a chronic condition that causes inflammation and sores in the lining of the colon and rectum, but can also affect the jejunum. Symptoms include diarrhea, abdominal pain, and bloody stools.
3. Jejunoileal bypass surgery: This is a type of bariatric surgery that involves rerouting the small intestine to reduce the amount of food that can be absorbed. While it can lead to weight loss, it can also cause nutrient deficiencies and other complications.
4. Jejunal tumors: These are growths that can occur in the jejunum, which can be benign or malignant. Symptoms include abdominal pain, bloating, and obstruction of the intestine.
5. Jejunal strictures: These are narrowing of the jejunum that can cause obstruction of food passage and lead to symptoms such as abdominal pain, nausea, and vomiting.
6. Jejunal inflammatory fibrosis: This is a condition where the jejunum becomes inflamed and scarred, leading to thickening of the intestinal walls and narrowing of the intestine. Symptoms include abdominal pain, diarrhea, and malabsorption.
7. Jejunal enteropathy: This is a condition where the jejunum becomes damaged, leading to symptoms such as diarrhea, abdominal pain, and weight loss. It can be caused by a variety of factors, including infection, inflammation, and autoimmune disorders.
8. Jejunal ulcers: These are open sores that can occur in the lining of the jejunum, often as a result of infection or inflammation. Symptoms include abdominal pain, nausea, and vomiting.
9. Jejunal ischemia: This is a condition where the blood supply to the jejunum is reduced, leading to damage to the intestinal tissue. Symptoms include abdominal pain, diarrhea, and rectal bleeding.
10. Jejunal cancer: This is a rare type of cancer that can occur in the jejunum. Symptoms include abdominal pain, weight loss, and rectal bleeding.
These are just a few examples of the many different conditions that can affect the jejunum. If you suspect that you or someone you know may have a condition affecting the jejunum, it is important to seek medical attention as soon as possible for proper diagnosis and treatment.
There are several types of intestinal obstruction, including:
1. Mechanical bowel obstruction: This type of obstruction is caused by a physical blockage in the intestine, such as adhesions or hernias.
2. Non-mechanical bowel obstruction: This type of obstruction is caused by a decrease in the diameter of the intestine, such as from inflammation or scarring.
3. Paralytic ileus: This type of obstruction is caused by a delay in the movement of food through the intestine, usually due to nerve damage or medication side effects.
4. Intestinal ischemia: This type of obstruction is caused by a decrease in blood flow to the intestine, which can lead to tissue damage and death.
Intestinal obstructions can be diagnosed through a variety of tests, including:
1. Abdominal X-rays: These can help identify any physical blockages in the intestine.
2. CT scans: These can provide more detailed images of the intestine and help identify any blockages or other issues.
3. Endoscopy: This involves inserting a flexible tube with a camera into the mouth and down into the intestine to visualize the inside of the intestine.
4. Biopsy: This involves removing a small sample of tissue from the intestine for examination under a microscope.
Treatment for intestinal obstructions depends on the underlying cause and severity of the blockage. Some common treatments include:
1. Fluid and electrolyte replacement: This can help restore hydration and electrolyte balance in the body.
2. Nasojejunal tube placement: A small tube may be inserted through the nose and into the jejunum to allow fluids and medications to pass through the blockage.
3. Surgery: In some cases, surgery may be necessary to remove the blockage or repair any damage to the intestine.
4. Medication: Depending on the underlying cause of the obstruction, medications such as antibiotics or anti-inflammatory drugs may be prescribed to help resolve the issue.
Preventing intestinal obstructions is often challenging, but some strategies can help reduce the risk. These include:
1. Avoiding foods that can cause blockages, such as nuts or seeds.
2. Eating a balanced diet and avoiding constipation.
3. Drinking plenty of fluids to stay hydrated.
4. Managing underlying medical conditions, such as inflammatory bowel disease or diabetes.
5. Avoiding medications that can cause constipation or other digestive problems.
The exact cause of ECN is not well understood, but it is believed to be associated with a combination of genetic and environmental factors, such as infections, medications, and underlying medical conditions like inflammatory bowel disease.
The symptoms of ECN can vary depending on the severity of the condition, but may include:
* Abdominal pain
* Diarrhea
* Fever
* Nausea and vomiting
* Fatigue
* Weight loss
* Loss of appetite
If you suspect that you or someone else may have ECN, it is important to seek medical attention immediately. A healthcare professional will perform a physical examination, take a medical history, and order diagnostic tests such as blood cultures, abdominal imaging (e.g., CT scan), and endoscopy to confirm the diagnosis and determine the extent of the condition.
Treatment of ECN typically involves supportive care to manage symptoms, address any underlying infections or other medical conditions, and prevent complications. This may include:
* Antibiotics to treat any underlying infections
* Pain management with medication
* Intravenous fluids and nutrition to prevent dehydration and malnutrition
* Surgical intervention to repair any perforations or remove damaged tissue
The prognosis for ECN can vary depending on the severity of the condition and the promptness and effectiveness of treatment. In general, early recognition and aggressive management of the condition can improve outcomes. However, the condition can be life-threatening and may result in long-term complications such as short bowel syndrome or chronic inflammatory bowel disease.
Prevention of ECN is not always possible, but good hand hygiene practices and proper use of personal protective equipment (PPE) can help reduce the risk of transmission. In addition, prompt recognition and treatment of underlying medical conditions can help prevent the development of ECN.
The primary symptoms of celiac disease include diarrhea, abdominal pain, fatigue, weight loss, and bloating. However, some people may not experience any symptoms at all, but can still develop complications if the disease is left untreated. These complications can include malnutrition, anemia, osteoporosis, and increased risk of other autoimmune disorders.
The exact cause of celiac disease is unknown, but it is believed to be triggered by a combination of genetic and environmental factors. The disease is more common in people with a family history of celiac disease or other autoimmune disorders. Diagnosis is typically made through a combination of blood tests and intestinal biopsy, and treatment involves a strict gluten-free diet.
Dietary management of celiac disease involves avoiding all sources of gluten, including wheat, barley, rye, and some processed foods that may contain hidden sources of these grains. In some cases, nutritional supplements may be necessary to ensure adequate intake of certain vitamins and minerals.
While there is no known cure for celiac disease, adherence to a strict gluten-free diet can effectively manage the condition and prevent long-term complications. With proper management, people with celiac disease can lead normal, healthy lives.
Symptoms of ileitis may include abdominal pain, diarrhea, fever, nausea, vomiting, and weight loss. Treatment for ileitis depends on the underlying cause and may include antibiotics, anti-inflammatory medications, and supportive care to manage symptoms.
In some cases, ileitis can lead to complications such as intestinal obstruction or perforation, which can be life-threatening. Prompt medical attention is essential to prevent these complications and ensure proper treatment.
The most common type of colitis is ulcerative colitis, which affects the rectum and lower part of the colon. The symptoms of ulcerative colitis can include:
* Diarrhea (which may be bloody)
* Abdominal pain and cramping
* Rectal bleeding
* Weight loss
* Fever
* Loss of appetite
* Nausea and vomiting
Microscopic colitis is another type of colitis that is characterized by inflammation in the colon, but without visible ulcers or bleeding. The symptoms of microscopic colitis are similar to those of ulcerative colitis, but may be less severe.
Other types of colitis include:
* Infantile colitis: This is a rare condition that affects babies and young children, and is characterized by diarrhea, fever, and vomiting.
* Isomorphic colitis: This is a rare condition that affects the colon and rectum, and is characterized by inflammation and symptoms similar to ulcerative colitis.
* Radiation colitis: This is a condition that occurs after radiation therapy to the pelvic area, and is characterized by inflammation and symptoms similar to ulcerative colitis.
* Ischemic colitis: This is a condition where there is a reduction in blood flow to the colon, which can lead to inflammation and symptoms such as abdominal pain and diarrhea.
The diagnosis of colitis typically involves a combination of physical examination, medical history, and diagnostic tests such as:
* Colonoscopy: This is a test that uses a flexible tube with a camera on the end to visualize the inside of the colon and rectum.
* Endoscopy: This is a test that uses a flexible tube with a camera on the end to visualize the inside of the esophagus, stomach, and duodenum.
* Stool tests: These are tests that analyze stool samples for signs of inflammation or infection.
* Blood tests: These are tests that analyze blood samples for signs of inflammation or infection.
* Biopsy: This is a test that involves taking a small sample of tissue from the colon and examining it under a microscope for signs of inflammation or infection.
Treatment for colitis depends on the underlying cause, but may include medications such as:
* Aminosalicylates: These are medications that help to reduce inflammation in the colon and relieve symptoms such as diarrhea and abdominal pain. Examples include sulfasalazine (Azulfidine) and mesalamine (Asacol).
* Corticosteroids: These are medications that help to reduce inflammation in the body. They may be used short-term to control acute flares of colitis, or long-term to maintain remission. Examples include prednisone and hydrocortisone.
* Immunomodulators: These are medications that help to suppress the immune system and reduce inflammation. Examples include azathioprine (Imuran) and mercaptopurine (Purinethol).
* Biologics: These are medications that target specific proteins involved in the inflammatory response. Examples include infliximab (Remicade) and adalimumab (Humira).
In addition to medication, lifestyle changes such as dietary modifications and stress management techniques may also be helpful in managing colitis symptoms. Surgery may be necessary in some cases where the colitis is severe or persistent, and involves removing damaged portions of the colon and rectum.
It's important to note that colitis can increase the risk of developing colon cancer, so regular screening for colon cancer is recommended for people with chronic colitis. Additionally, people with colitis may be more susceptible to other health problems such as osteoporosis, osteopenia, and liver disease, so it's important to work closely with a healthcare provider to monitor for these conditions and take steps to prevent them.
Body weight is an important health indicator, as it can affect an individual's risk for certain medical conditions, such as obesity, diabetes, and cardiovascular disease. Maintaining a healthy body weight is essential for overall health and well-being, and there are many ways to do so, including a balanced diet, regular exercise, and other lifestyle changes.
There are several ways to measure body weight, including:
1. Scale: This is the most common method of measuring body weight, and it involves standing on a scale that displays the individual's weight in kg or lb.
2. Body fat calipers: These are used to measure body fat percentage by pinching the skin at specific points on the body.
3. Skinfold measurements: This method involves measuring the thickness of the skin folds at specific points on the body to estimate body fat percentage.
4. Bioelectrical impedance analysis (BIA): This is a non-invasive method that uses electrical impulses to measure body fat percentage.
5. Dual-energy X-ray absorptiometry (DXA): This is a more accurate method of measuring body composition, including bone density and body fat percentage.
It's important to note that body weight can fluctuate throughout the day due to factors such as water retention, so it's best to measure body weight at the same time each day for the most accurate results. Additionally, it's important to use a reliable scale or measuring tool to ensure accurate measurements.
Crohn disease can occur in any part of the GI tract, from the mouth to the anus, but it most commonly affects the ileum (the last portion of the small intestine) and the colon. The inflammation caused by Crohn disease can lead to the formation of scar tissue, which can cause narrowing or blockages in the intestines. This can lead to complications such as bowel obstruction or abscesses.
The exact cause of Crohn disease is not known, but it is believed to be an autoimmune disorder, meaning that the immune system mistakenly attacks healthy tissue in the GI tract. Genetic factors and environmental triggers such as smoking and diet also play a role in the development of the disease.
There is no cure for Crohn disease, but various treatments can help manage symptoms and prevent complications. These may include medications such as anti-inflammatory drugs, immunosuppressants, and biologics, as well as lifestyle changes such as dietary modifications and stress management techniques. In severe cases, surgery may be necessary to remove damaged portions of the GI tract.
Crohn disease can have a significant impact on quality of life, and it is important for individuals with the condition to work closely with their healthcare provider to manage their symptoms and prevent complications. With proper treatment and self-care, many people with Crohn disease are able to lead active and fulfilling lives.
Example sentences:
1) The patient was diagnosed with enterocolitis after presenting with severe abdominal pain and diarrhea.
2) The doctor suspected enterocolitis based on the patient's symptoms, but further testing was needed to confirm the diagnosis.
3) Enterocolitis can be a serious condition that requires prompt medical attention to prevent complications such as dehydration and electrolyte imbalances.
Some common types of intestinal diseases, parasitic include:
1. Amoebiasis: This is an infection caused by the amoeba Entamoeba histolytica, which can cause diarrhea, abdominal pain, and fever.
2. Giardiasis: This is an infection caused by the parasite Giardia duodenalis, which can cause diarrhea, abdominal cramps, and weight loss.
3. Cryptosporidiosis: This is an infection caused by the parasite Cryptosporidium parvum, which can cause diarrhea, abdominal pain, and fever.
4. Isosporiasis: This is an infection caused by the parasite Isospora belli, which can cause diarrhea, abdominal cramps, and weight loss.
5. Tapeworm infections: These are infections caused by tapeworms, such as Taenia saginata (beef tapeworm) and Dipylidium caninum (dog tapeworm), which can cause abdominal pain, diarrhea, and weight loss.
6. Strongyloidiasis: This is an infection caused by the parasite Strongyloides stercoralis, which can cause diarrhea, abdominal pain, and fatigue.
Intestinal diseases, parasitic can be diagnosed through a combination of physical examination, medical history, and laboratory tests such as stool samples or blood tests. Treatment depends on the specific type of infection and may include antiparasitic medications, anti-diarrheal medications, and supportive care to manage symptoms.
A disease that affects pigs, including viral, bacterial, and parasitic infections, as well as genetic disorders and nutritional deficiencies. Some common swine diseases include:
1. Porcine Reproductive and Respiratory Syndrome (PRRS): A highly contagious viral disease that can cause reproductive failure, respiratory problems, and death.
2. Swine Influenza: A viral infection similar to human influenza, which can cause fever, coughing, and pneumonia in pigs.
3. Erysipelas: A bacterial infection that causes high fever, loss of appetite, and skin lesions in pigs.
4. Actinobacillosis: A bacterial infection that can cause pneumonia, arthritis, and abscesses in pigs.
5. Parasitic infections: Such as gastrointestinal parasites like roundworms and tapeworms, which can cause diarrhea, anemia, and weight loss in pigs.
6. Scrapie: A degenerative neurological disorder that affects pigs and other animals, causing confusion, aggression, and eventually death.
7. Nutritional deficiencies: Such as a lack of vitamin E or selenium, which can cause a range of health problems in pigs, including muscular dystrophy and anemia.
8. Genetic disorders: Such as achondroplasia, a condition that causes dwarfism and deformities in pigs.
9. Environmental diseases: Such as heat stress, which can cause a range of health problems in pigs, including respiratory distress and death.
It's important to note that many swine diseases have similar symptoms, making accurate diagnosis by a veterinarian essential for effective treatment and control.
The most common types of trematode infections include:
1. Schistosomiasis: This is a parasitic disease caused by Schistosoma worms that affects over 200 million people worldwide, primarily in developing countries. It is spread through contact with contaminated freshwater snails.
2. Fascioliasis (also known as liver fluke): This is a parasitic disease caused by Fasciola worms that affects humans and various animals, including sheep, cattle, and pigs. It is spread through consumption of contaminated water or food.
3. Clonorchiasis: This is a parasitic disease caused by Clonorchis sinensis worms that affects humans in parts of Asia, particularly in China and Korea. It is spread through consumption of raw or undercooked fish.
4. Opisthorchiasis: This is a parasitic disease caused by Opisthorchis viverrini worms that affects humans in parts of Southeast Asia, particularly in Thailand and Laos. It is spread through consumption of raw or undercooked fish.
The symptoms of trematode infections vary depending on the type of parasite and the organs affected, but they can include:
* Abdominal pain
* Diarrhea
* Fatigue
* Weakness
* Loss of appetite
* Nausea and vomiting
* Jaundice (yellowing of the skin and eyes)
* Inflammation of the liver, lung, or other organs
Trematode infections can be diagnosed through various techniques, including:
1. Microscopic examination of stool samples for parasite eggs or larvae.
2. Serological tests such as ELISA or immunochromatography to detect antibodies against the parasite in the blood.
3. Imaging techniques such as ultrasound, CT scan, or MRI to visualize the parasites or their effects on organs.
4. Endoscopy to examine the digestive tract for parasites or inflammation.
Treatment of trematode infections depends on the type of parasite and the severity of the infection, but it often involves anti-parasitic drugs such as praziquantel, triclabendazole, or oxfendazole, which are effective against most trematodes. In severe cases, hospitalization may be required to manage complications such as liver inflammation or respiratory failure. Prevention measures include:
1. Avoiding consumption of raw or undercooked fish and other aquatic animals.
2. Properly cooking fish and other seafood before eating them.
3. Using clean water for drinking, cooking, and washing.
4. Avoiding contact with contaminated water or snails that may carry trematodes.
5. Implementing sanitation and hygiene measures in areas where trematode infections are common.
Trematodes are a diverse group of parasites that can infect humans and other animals, causing a range of diseases with varying severity. Diagnosis is based on serological or imaging techniques, and treatment involves anti-parasitic drugs. Prevention measures include avoiding raw or undercooked seafood, properly cooking fish and other seafood, using clean water, and implementing sanitation and hygiene measures in areas where trematode infections are common.
1. Crohn's disease: A chronic inflammatory condition that can affect any part of the gastrointestinal tract, but most commonly affects the ileum.
2. Ulcerative colitis: A chronic inflammatory condition that affects the large intestine and rectum, but can also affect the ileum.
3. Ileal tumors: Such as carcinoid tumors, lymphoma, and sarcomas.
4. Ileal polyps: Growths of abnormal tissue in the ileum that can cause bleeding, obstruction, or cancer.
5. Inflammatory bowel disease (IBD): A group of chronic conditions, including Crohn's disease and ulcerative colitis, that cause inflammation in the digestive tract.
6. Ileal strictures: Narrowing of the ileum that can cause obstruction and blockage of food passage.
7. Ileal dilatation: Expansion of the ileum beyond its normal size, which can cause abdominal pain and discomfort.
8. Ileal ischemia: Reduced blood flow to the ileum, which can cause damage and inflammation.
9. Ileal infections: Such as bacterial or viral infections that can cause inflammation and damage to the ileum.
10. Ileal varices: Enlarged veins in the ileum that can cause bleeding and other complications.
These are some of the common ileal diseases, but there may be others depending on the individual case and specific symptoms. It is important to seek medical attention if you experience any persistent or severe abdominal symptoms to get an accurate diagnosis and appropriate treatment.
Crohn's disease can affect any part of the GI tract, from the mouth to the anus, and causes symptoms such as abdominal pain, diarrhea, fatigue, and weight loss. Ulcerative colitis primarily affects the colon and rectum and causes symptoms such as bloody stools, abdominal pain, and weight loss.
Both Crohn's disease and ulcerative colitis are chronic conditions, meaning they cannot be cured but can be managed with medication and lifestyle changes. Treatment options for IBD include anti-inflammatory medications, immunosuppressants, and biologics. In severe cases, surgery may be necessary to remove damaged portions of the GI tract.
There is no known cause of IBD, although genetics, environmental factors, and an abnormal immune response are thought to play a role. The condition can have a significant impact on quality of life, particularly if left untreated or poorly managed. Complications of IBD include malnutrition, osteoporosis, and increased risk of colon cancer.
Preventing and managing IBD requires a comprehensive approach that includes medication, dietary changes, stress management, and regular follow-up with a healthcare provider. With proper treatment and lifestyle modifications, many people with IBD are able to manage their symptoms and lead active, fulfilling lives.
Reperfusion injury can cause inflammation, cell death, and impaired function in the affected tissue or organ. The severity of reperfusion injury can vary depending on the duration and severity of the initial ischemic event, as well as the promptness and effectiveness of treatment to restore blood flow.
Reperfusion injury can be a complicating factor in various medical conditions, including:
1. Myocardial infarction (heart attack): Reperfusion injury can occur when blood flow is restored to the heart muscle after a heart attack, leading to inflammation and cell death.
2. Stroke: Reperfusion injury can occur when blood flow is restored to the brain after an ischemic stroke, leading to inflammation and damage to brain tissue.
3. Organ transplantation: Reperfusion injury can occur when a transplanted organ is subjected to ischemia during harvesting or preservation, and then reperfused with blood.
4. Peripheral arterial disease: Reperfusion injury can occur when blood flow is restored to a previously occluded peripheral artery, leading to inflammation and damage to the affected tissue.
Treatment of reperfusion injury often involves medications to reduce inflammation and oxidative stress, as well as supportive care to manage symptoms and prevent further complications. In some cases, experimental therapies such as stem cell transplantation or gene therapy may be used to promote tissue repair and regeneration.
There are many different types of helminths that can infect animals, including:
* Roundworms (Toxocara canis, Toxocara cati)
* Hookworms (Ancylostoma caninum, Ancylostoma braziliense)
* Whipworms (Trichuris vulpis)
* Tapeworms (Taenia pisiformis, Taenia serialis)
* Liver flukes (Fasciola hepatica)
* Intestinal flukes (Fasciola gigantica)
Animals can become infected with helminths through a variety of means, including:
* Ingestion of contaminated food or water
* Contact with infected animals or their feces
* Insect vectors, such as mosquitoes or fleas
The symptoms of helminthiasis can vary depending on the type of worm and the severity of the infection. Some common symptoms include:
* Diarrhea
* Vomiting
* Abdominal pain
* Weight loss
* Anemia
* Inflammation of various organs, such as the liver or kidneys
In severe cases, helminthiasis can lead to more serious complications, such as intestinal blockages or abscesses.
Diagnosis of helminthiasis typically involves a combination of physical examination, laboratory tests, and imaging studies. Treatment usually involves the use of antiparasitic drugs to kill the worms, and may also include supportive care to manage symptoms and prevent complications.
Prevention of helminthiasis is important for both animal and human health, and can be achieved through a variety of measures, including:
* Regular deworming of animals
* Proper disposal of animal feces
* Safe handling and cooking of food
* Avoiding contact with contaminated water or soil
* Using insecticides to control vectors, such as mosquitoes and fleas.
There are several types of malabsorption syndromes, including:
1. Celiac disease: An autoimmune disorder that damages the lining of the small intestine and interferes with nutrient absorption.
2. Crohn's disease: An inflammatory bowel disease that can damage the small intestine and lead to malabsorption.
3. Whipple's disease: A bacterial infection that causes inflammation and damage to the small intestine.
4. Giant cell enteropathy: An immune-mediated disorder that damages the small intestine and interferes with nutrient absorption.
5. Postoperative malabsorption: Malabsorption that occurs after surgery on the small intestine.
6. Pancreatic insufficiency: A condition in which the pancreas is unable to produce enough digestive enzymes to break down food properly.
7. Bacterial overgrowth: An overgrowth of bacteria in the small intestine can interfere with nutrient absorption.
8. Food allergies or intolerances: Certain foods can cause an immune response or irritation to the small intestine, leading to malabsorption.
The symptoms of malabsorption syndromes vary depending on the specific disorder and the severity of the condition. Common symptoms include diarrhea, abdominal pain, weight loss, and nutrient deficiencies. Treatment depends on the underlying cause of the malabsorption and may involve dietary changes, medication, or surgery.
Here are some common types of E. coli infections:
1. Urinary tract infections (UTIs): E. coli is a leading cause of UTIs, which occur when bacteria enter the urinary tract and cause inflammation. Symptoms include frequent urination, burning during urination, and cloudy or strong-smelling urine.
2. Diarrheal infections: E. coli can cause diarrhea, abdominal cramps, and fever if consumed through contaminated food or water. In severe cases, this type of infection can lead to dehydration and even death, particularly in young children and the elderly.
3. Septicemia (bloodstream infections): If E. coli bacteria enter the bloodstream, they can cause septicemia, a life-threatening condition that requires immediate medical attention. Symptoms include fever, chills, rapid heart rate, and low blood pressure.
4. Meningitis: In rare cases, E. coli infections can spread to the meninges, the protective membranes covering the brain and spinal cord, causing meningitis. This is a serious condition that requires prompt treatment with antibiotics and supportive care.
5. Hemolytic-uremic syndrome (HUS): E. coli infections can sometimes cause HUS, a condition where the bacteria destroy red blood cells, leading to anemia, kidney failure, and other complications. HUS is most common in young children and can be fatal if not treated promptly.
Preventing E. coli infections primarily involves practicing good hygiene, such as washing hands regularly, especially after using the bathroom or before handling food. It's also essential to cook meat thoroughly, especially ground beef, to avoid cross-contamination with other foods. Avoiding unpasteurized dairy products and drinking contaminated water can also help prevent E. coli infections.
If you suspect an E. coli infection, seek medical attention immediately. Your healthcare provider may perform a urine test or a stool culture to confirm the diagnosis and determine the appropriate treatment. In mild cases, symptoms may resolve on their own within a few days, but antibiotics may be necessary for more severe infections. It's essential to stay hydrated and follow your healthcare provider's recommendations to ensure a full recovery.
There are several types of ulcers, including:
1. Peptic ulcer: A type of ulcer that occurs in the lining of the stomach or duodenum (the first part of the small intestine). Peptic ulcers are caused by excess acid production and are often associated with stress, spicy foods, and certain medications.
2. Stomal ulcer: A type of ulcer that occurs in the stoma (the opening) of a surgically created ostomy (a procedure that creates an artificial opening in the abdominal wall).
3. Pressure ulcer: A type of ulcer that occurs as a result of prolonged pressure on the skin, often seen in people who are bedridden or have mobility issues.
4. Venous ulcer: A type of ulcer that occurs on the legs and is caused by poor blood flow and increased pressure in the veins.
5. Diabetic foot ulcer: A type of ulcer that occurs on the feet of people with diabetes, often as a result of nerve damage (neuropathy) and poor blood flow.
The symptoms of an ulcer can vary depending on its location and severity, but may include:
* Pain or discomfort in the affected area
* Redness and swelling around the ulcer
* Discharge or pus from the ulcer
* Fever or chills
* Difficulty healing
Treatment for an ulcer will depend on its cause and severity, but may include:
* Antibiotics to treat any underlying infections
* Medications to reduce acid production or protect the stomach lining
* Wound care and dressing changes to promote healing
* Surgery to close the ulcer or remove any dead tissue
* Changes to diet and lifestyle to manage underlying conditions such as diabetes or high blood pressure.
The symptoms of short bowel syndrome can vary depending on the severity of the condition and may include:
* Diarrhea
* Abdominal pain
* Nausea and vomiting
* Weight loss
* Fatigue
* Dehydration
* Malnutrition
Treatment for short bowel syndrome typically involves a combination of dietary modifications, medications, and supplements to help manage symptoms and improve nutrient absorption. In some cases, intravenous feeding may be necessary to ensure adequate nutrition.
Short bowel syndrome can be caused by a variety of factors, including:
* Intestinal surgery
* Inflammatory bowel disease (such as Crohn's disease or ulcerative colitis)
* Infections (such as Clostridium difficile or viral infections)
* Radiation therapy
* Trauma to the abdomen
* Congenital conditions (such as short gut syndrome)
Overall, short bowel syndrome can have a significant impact on quality of life and can be challenging to manage. However, with proper treatment and support, it is possible for individuals with this condition to lead active and fulfilling lives.
There are several types of colonic neoplasms, including:
1. Adenomas: These are benign growths that are usually precursors to colorectal cancer.
2. Carcinomas: These are malignant tumors that arise from the epithelial lining of the colon.
3. Sarcomas: These are rare malignant tumors that arise from the connective tissue of the colon.
4. Lymphomas: These are cancers of the immune system that can affect the colon.
Colonic neoplasms can cause a variety of symptoms, including bleeding, abdominal pain, and changes in bowel habits. They are often diagnosed through a combination of medical imaging tests (such as colonoscopy or CT scan) and biopsy. Treatment for colonic neoplasms depends on the type and stage of the tumor, and may include surgery, chemotherapy, and/or radiation therapy.
Overall, colonic neoplasms are a common condition that can have serious consequences if left untreated. It is important for individuals to be aware of their risk factors and to undergo regular screening for colon cancer to help detect and treat any abnormal growths or tumors in the colon.
Types of Gastrointestinal Diseases:
1. Irritable Bowel Syndrome (IBS): A common condition characterized by abdominal pain, bloating, and changes in bowel movements.
2. Inflammatory Bowel Disease (IBD): A group of chronic conditions that cause inflammation in the digestive tract, including Crohn's disease and ulcerative colitis.
3. Gastroesophageal Reflux Disease (GERD): A condition in which stomach acid flows back into the esophagus, causing heartburn and other symptoms.
4. Peptic Ulcer Disease: A condition characterized by ulcers in the lining of the stomach or duodenum.
5. Diverticulitis: A condition in which small pouches form in the wall of the colon and become inflamed.
6. Gastritis: Inflammation of the stomach lining, often caused by infection or excessive alcohol consumption.
7. Esophagitis: Inflammation of the esophagus, often caused by acid reflux or infection.
8. Rectal Bleeding: Hemorrhage from the rectum, which can be a symptom of various conditions such as hemorrhoids, anal fissures, or inflammatory bowel disease.
9. Functional Dyspepsia: A condition characterized by recurring symptoms of epigastric pain, bloating, nausea, and belching.
10. Celiac Disease: An autoimmune disorder that causes the immune system to react to gluten, leading to inflammation and damage in the small intestine.
Causes of Gastrointestinal Diseases:
1. Infection: Viral, bacterial, or parasitic infections can cause gastrointestinal diseases.
2. Autoimmune Disorders: Conditions such as Crohn's disease and ulcerative colitis occur when the immune system mistakenly attacks healthy tissue in the GI tract.
3. Diet: Consuming a diet high in processed foods, sugar, and unhealthy fats can contribute to gastrointestinal diseases.
4. Genetics: Certain genetic factors can increase the risk of developing certain gastrointestinal diseases.
5. Lifestyle Factors: Smoking, excessive alcohol consumption, stress, and lack of physical activity can all contribute to gastrointestinal diseases.
6. Radiation Therapy: Exposure to radiation therapy can damage the GI tract and increase the risk of developing certain gastrointestinal diseases.
7. Medications: Certain medications such as nonsteroidal anti-inflammatory drugs (NSAIDs) and corticosteroids can cause gastrointestinal side effects.
The symptoms of cholera include:
1. Diarrhea: Cholera causes profuse, watery diarrhea that can last for several days.
2. Dehydration: The loss of fluids and electrolytes due to diarrhea can lead to severe dehydration, which can be life-threatening if not treated promptly.
3. Nausea and vomiting: Cholera patients may experience nausea and vomiting, especially in the early stages of the disease.
4. Abdominal cramps: The abdomen may become tender and painful due to the inflammation caused by the bacteria.
5. Low-grade fever: Some patients with cholera may experience a mild fever, typically less than 102°F (39°C).
Cholera is spread through the fecal-oral route, which means that it is transmitted when someone ingests food or water contaminated with the bacteria. The disease can also be spread by direct contact with infected fecal matter, such as through poor hygiene practices or inadequate waste disposal.
There are several ways to diagnose cholera, including:
1. Stool test: A stool sample can be tested for the presence of Vibrio cholerae using a microscope or a rapid diagnostic test (RDT).
2. Blood test: A blood test can detect the presence of antibodies against Vibrio cholerae, which can indicate that the patient has been infected with the bacteria.
3. Physical examination: A healthcare provider may perform a physical examination to look for signs of dehydration and other symptoms of cholera.
Treatment of cholera typically involves replacing lost fluids and electrolytes through oral rehydration therapy (ORT) or intravenous fluids. Antibiotics may also be given to shorten the duration of diarrhea and reduce the risk of complications. In severe cases, hospitalization may be necessary to provide more intensive treatment.
Prevention of cholera involves maintaining good hygiene practices, such as washing hands with soap and water, and avoiding consumption of contaminated food and water. Vaccines are also available to protect against cholera, particularly for people living in areas where the disease is common.
In conclusion, cholera is a highly infectious disease that can cause severe dehydration and even death if left untreated. Early diagnosis and treatment are critical to preventing complications and reducing the risk of transmission. Prevention measures such as vaccination and good hygiene practices can also help control the spread of the disease.
The term "Salmonella Infections, Animal" is used to distinguish these infections from Salmonella infections that are caused by contaminated food or water, which are referred to as "Salmonella Infections, Human."
Starvation is a condition where an individual's body does not receive enough nutrients to maintain proper bodily functions and growth. It can be caused by a lack of access to food, poverty, poor nutrition, or other factors that prevent the intake of sufficient calories and essential nutrients. Starvation can lead to severe health consequences, including weight loss, weakness, fatigue, and even death.
Types of Starvation:
There are several types of starvation, each with different causes and effects. These include:
1. Acute starvation: This occurs when an individual suddenly stops eating or has a limited access to food for a short period of time.
2. Chronic starvation: This occurs when an individual consistently does not consume enough calories and nutrients over a longer period of time, leading to gradual weight loss and other health problems.
3. Malnutrition starvation: This occurs when an individual's diet is deficient in essential nutrients, leading to malnutrition and other health problems.
4. Marasmus: This is a severe form of starvation that occurs in children, characterized by extreme weight loss, weakness, and wasting of muscles and organs.
5. Kwashiorkor: This is a form of malnutrition caused by a diet lacking in protein, leading to edema, diarrhea, and other health problems.
Effects of Starvation on the Body:
Starvation can have severe effects on the body, including:
1. Weight loss: Starvation causes weight loss, which can lead to a decrease in muscle mass and a loss of essential nutrients.
2. Fatigue: Starvation can cause fatigue, weakness, and a lack of energy, making it difficult to perform daily activities.
3. Weakened immune system: Starvation can weaken the immune system, making an individual more susceptible to illnesses and infections.
4. Nutrient deficiencies: Starvation can lead to a deficiency of essential nutrients, including vitamins and minerals, which can cause a range of health problems.
5. Increased risk of disease: Starvation can increase the risk of diseases such as tuberculosis, pellagra, and other infections.
6. Mental health issues: Starvation can lead to mental health issues such as depression, anxiety, and irritability.
7. Reproductive problems: Starvation can cause reproductive problems, including infertility and miscarriage.
8. Hair loss: Starvation can cause hair loss, which can be a sign of malnutrition.
9. Skin problems: Starvation can cause skin problems, such as dryness, irritation, and infections.
10. Increased risk of death: Starvation can lead to increased risk of death, especially in children and the elderly.
It is important to note that these effects can be reversed with proper nutrition and care. If you or someone you know is experiencing starvation, it is essential to seek medical attention immediately.
UC can be challenging to diagnose and treat, and there is no known cure. However, with proper management, it is possible for people with UC to experience long periods of remission and improve their quality of life. Treatment options include medications such as aminosalicylates, corticosteroids, and immunomodulators, as well as surgery in severe cases.
It's important for individuals with UC to work closely with their healthcare provider to develop a personalized treatment plan that takes into account their specific symptoms and needs. With the right treatment and support, many people with UC are able to manage their symptoms and lead active, fulfilling lives.
Stuffed intestines
Vasa recta (intestines)
Braised intestines in brown sauce
Large intestine
Small intestine
Intestine cancer
The Intestine
Intestine transplantation
Small intestine cancer
Intestine-on-a-chip
Small intestine neuroendocrine tumor
Small intestine (Chinese medicine)
Large intestine (Chinese medicine)
Multifocal stenosing ulceration of the small intestine
Rhynchotermes
Gopchang
List of kanji by concept
Yiddish words used in English
List of fictional vegetarian characters
Battle of Humera
Alexander Polycleitos Cawadias
Gopchang-jeongol
Murray's law
The Canon of Medicine
East Asian finless porpoise
Solitary (TV series)
Indo-Pacific finless porpoise
Vaginal evisceration
Charles II of Spain
Kareem Mohamed Abu-Elmagd
Eberhard Eggers. Homage to Intestine. (1964) | MoMA
Infant intestines: MedlinePlus Medical Encyclopedia Image
Intestines Archives - Better Health - Better Health
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Anatomy of the Intestines and Microvilli | Biology | JoVE
intestines | Memoir Revolution
CPT® Code - Surgical Procedures on the Intestines (Except Rectum) 44005-44799 - Codify by AAPC
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This woman had wire removed from her small intestine 10 years after her braces were removed - HelloGigglesHelloGiggles
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Stomach5
- The gastrointestinal tract (or stomach and intestines) digests food and makes it ready to be absorbed into the bloodstream. (cdc.gov)
- The digestive system consists of the oral cavity (mouth), esophagus, stomach, gut (small and large intestines), and rectum. (cdc.gov)
- Gastroenterologists specialize in treating conditions relating to the digestive system, including the stomach, intestines and colon. (medibid.com)
- Through the stomach, the bacteria reach the small intestine. (medscape.com)
- Paraquat causes direct damage when it comes into contact with the lining of the mouth, stomach, or intestines. (cdc.gov)
Liver1
- The smaller volume of intestine, lower amount of CYP, and higher Km for TCP in the enterocyte microsomes, resulted in a lower catalytic efficiency (2 and 62 times) than in liver for oxon and TCP. (cdc.gov)
Rectum1
- The Current Procedural Terminology (CPT) code range for Surgical Procedures on the Intestines (Except Rectum) 44005-44799 is a medical code set maintained by the American Medical Association. (aapc.com)
Intestinal2
- jostling of internal organs, relative intestinal ischemia (decreased oxygen getting to the intestines as blood is diverted to the muscles for work and to the skin to cool the body), dehydration, and adrenaline-related anxiety/stress hormones. (getbetterhealth.com)
- We will also translate our murine studies tohuman intestinal organoids to provide mechanistic insights on the role of HDAC3 and epigenetics in host-microbeinteractions in human intestine. (usda.gov)
Small1
- Pantomics Array Description: Small intestine cancer tissue array, containing 97 cases of small intestine cancer, 5 cases of. (delos.info)
Immune2
- Immune functions in the mouse intestine differ by segment. (the-scientist.com)
- We will (i) interrogate how active regulation of IECs by HDAC3directs tissue-resident lymphocyte dynamics and test how this epithelial-immune cell relationship ismechanistically controlled, (ii) directly examine how the microbiota promote epithelial antimicrobial secretionduring infection, and (iii) employ reporter mice to define whether epigenetic regulation of stem cells mediateshow the microbiota sustains long-term defense in the intestine. (usda.gov)
Large1
- They work by increasing the amount of water in your large intestine . (synonym.com)
Species1
- Colin Basler] Many different bird species can harbor Salmonella in their intestines. (cdc.gov)
Case1
- Sometimes, the tip of the endoscopy tube can get stuck, and as more tube is fed in, it causes the intestine to distend, which best case is painful and worst case can cause serious internal injuries. (ieee.org)
Work1
- This work will uncover novel mechanisms for how IECs integrate microbialsignals to instruct intestine-intrinsic immunity and guide design of next generation therapeutics that canepigenetically prime the intestine to effectively defend against enteric infections. (usda.gov)
Time1
- It is also important for backyard poultry owners to know that Salmonella can live in the intestines of chickens and can survive on feathers for some time. (cdc.gov)
Health2
- Pediatric intestine transplant cost: Analysis of the Pediatric Health Information System database. (bvsalud.org)
- We performed a cross-sectional observational study of pediatric intestine transplant recipients from 2004 through 2020, utilizing the Pediatric Health Information System database. (bvsalud.org)
Amount1
- The food inspection bureau found out that many of the restaurants were not properly cleaning the intestines so there was an excess amount of fecal matter in the food. (seouleats.com)
Blood1
- This is blood sausage and intestine stir fry. (zenkimchi.com)
Baby2
- And when their baby boy was born with his intestines on the outside of his body, a miracle is just what they got! (godvine.com)
- The couple's precious baby boy -- Alec Ezra Romo -- was born three weeks early, with his intestines on the outside of his body. (godvine.com)
Children1
- We aimed to evaluate costs from transplant to discharge in children who had undergone intestine transplant . (bvsalud.org)
Good2
- The reason why a robot like Endoculus is necessary (or at least a good idea) is that trying to stuff a semi-rigid endoscopy tube into the semi-floppy tube that is your intestine doesn't always go smoothly. (ieee.org)
- Grilled intestines, to us, taste like good old fashioned breakfast bacon. (zenkimchi.com)
Thought1
- At first they thought it was a fish bone…and it had pierced her intestine in multiple areas, causing a condition that required immediate surgery. (hellogiggles.com)
Duodenum2
- The small intestine (small bowel) lies between the stomach and the large intestine (large bowel) and includes the duodenum, jejunum, and ileum. (medscape.com)
- The small intestine is differentiated from the large intestine by the presence of a mesentery (exceptions being no mesentery in the duodenum, and presence of a mesentery in the transverse and sigmoid colons) and the absence of tenia coli and appendices epiploicae, which are present in the colon. (medscape.com)
Jejunum1
- The jejunum constitutes about two fifths of the proximal small intestine and the ileum makes the distal three fifths. (medscape.com)
Transplant3
- As survival rates increase for intestine transplant recipients, with many patients having an extended survival of greater than 5 years, quality-of-life issues are beginning to be examined. (medscape.com)
- [ 105 ] Quality of life improved over time in adult intestine transplant recipients who were evaluated at a mean time of 2.7 and 5.3 years after transplantation. (medscape.com)
- Assessing quality of life in intestine transplant recipients is challenging due to significant patient variability with respect to underlying disease, the postoperative course, long-term complications, and psychosocial factors. (medscape.com)
Cecum3
- The large intestine develops partly from the midgut (from cecum to distal transverse colon), the hindgut (from distal transverse colon to dentate line in anorectum), and proctodeum (below the dentate line). (medscape.com)
- The cecum (the widest part of large intestine) leads to the ascending (right) colon, which ascends vertically from right iliac fossa through the right lumbar region into right hypochondrium under the liver. (medscape.com)
- The ileum continues into the large intestine (cecum) at the ileocecal junction. (medscape.com)
Rectum1
- The digestive system consists of the oral cavity (mouth), esophagus, stomach, gut (small and large intestines), and rectum. (cdc.gov)
Midgut2
- Embryologically, the small intestine develops mainly from the midgut, with the superior mesenteric artery (SMA) as its artery. (medscape.com)
- The midgut also gives rise to the proximal large intestine (up to the proximal two thirds of the transverse colon). (medscape.com)
Fluid2
- The intestines are responsible for the majority of fluid absorption. (medlineplus.gov)
- Collectively, microvilli make up an absorptive surface, called the brush border, where digested nutrients in the fluid passing through the intestine can enter cells via transport channels. (nih.gov)
Human2
Length1
- Psychiatric and psychosocial problems affecting quality of life following intestine transplantation are a function of severity of disease, duration of preoperative TPN, length of the waiting period, and prolonged postoperative course, and vary inversely with available social support. (medscape.com)