AnatomyOrganismsDiseasesChemicals and DrugsAnalytical, Diagnostic and Therapeutic Techniques and EquipmentPhenomena and ProcessesHealth CareGeographicals
HypopigmentationPigmentation DisordersAlbinism, OcularAlbinism, OculocutaneousHair ColorHermanski-Pudlak SyndromeMelanosomesEye ColorHyperpigmentationSkin PigmentationVitiligoAdaptor Protein Complex beta SubunitsMonophenol MonooxygenasePlatelet Storage Pool DeficiencyWaardenburg SyndromeMelanocytesNystagmus, CongenitalPigmentationAlbinismSOXE Transcription FactorsMelaninsMicrophthalmia-Associated Transcription FactorPrader-Willi SyndromeChromosomes, Human, Pair 15Pigment Epithelium of EyeHairPedigreeSyndromeSkin DiseasesGenome-Wide Association StudyPolymorphism, Single NucleotideCattleIridaceaeChediak-Higashi SyndromeLeishmania donovaniLeishmaniasis, VisceralLeishmaniasis, CutaneousIndiaPhlebotomusAntiprotozoal AgentsAntimony Sodium Gluconate
Hypopigmentation
A condition caused by a deficiency or a loss of melanin pigmentation in the epidermis, also known as hypomelanosis. Hypopigmentation can be localized or generalized, and may result from genetic defects, trauma, inflammation, or infections.
Pigmentation Disorders
Albinism, Ocular
Albinism affecting the eye in which pigment of the hair and skin is normal or only slightly diluted. The classic type is X-linked (Nettleship-Falls), but an autosomal recessive form also exists. Ocular abnormalities may include reduced pigmentation of the iris, nystagmus, photophobia, strabismus, and decreased visual acuity.
Albinism, Oculocutaneous
Heterogeneous group of autosomal recessive disorders comprising at least four recognized types, all having in common varying degrees of hypopigmentation of the skin, hair, and eyes. The two most common are the tyrosinase-positive and tyrosinase-negative types.
Hair Color
Color of hair or fur.
Hermanski-Pudlak Syndrome
Syndrome characterized by the triad of oculocutaneous albinism (ALBINISM, OCULOCUTANEOUS); PLATELET STORAGE POOL DEFICIENCY; and lysosomal accumulation of ceroid lipofuscin.
Melanosomes
Melanin-containing organelles found in melanocytes and melanophores.
Eye Color
Color of the iris.
Hyperpigmentation
Excessive pigmentation of the skin, usually as a result of increased epidermal or dermal melanin pigmentation, hypermelanosis. Hyperpigmentation can be localized or generalized. The condition may arise from exposure to light, chemicals or other substances, or from a primary metabolic imbalance.
Skin Pigmentation
Coloration of the skin.
Vitiligo
A disorder consisting of areas of macular depigmentation, commonly on extensor aspects of extremities, on the face or neck, and in skin folds. Age of onset is often in young adulthood and the condition tends to progress gradually with lesions enlarging and extending until a quiescent state is reached.
Adaptor Protein Complex beta Subunits
A family of large adaptin protein complex subunits of approximately 90-130 kDa in size.
Monophenol Monooxygenase
An enzyme of the oxidoreductase class that catalyzes the reaction between L-tyrosine, L-dopa, and oxygen to yield L-dopa, dopaquinone, and water. It is a copper protein that acts also on catechols, catalyzing some of the same reactions as CATECHOL OXIDASE. EC 1.14.18.1.
Platelet Storage Pool Deficiency
Disorder characterized by a decrease or lack of platelet dense bodies in which the releasable pool of adenine nucleotides and 5HT are normally stored.
Waardenburg Syndrome
Rare, autosomal dominant disease with variable penetrance and several known clinical types. Characteristics may include depigmentation of the hair and skin, congenital deafness, heterochromia iridis, medial eyebrow hyperplasia, hypertrophy of the nasal root, and especially dystopia canthorum. The underlying cause may be defective development of the neural crest (neurocristopathy). Waardenburg's syndrome may be closely related to piebaldism. Klein-Waardenburg Syndrome refers to a disorder that also includes upper limb abnormalities.
Melanocytes
Mammalian pigment cells that produce MELANINS, pigments found mainly in the EPIDERMIS, but also in the eyes and the hair, by a process called melanogenesis. Coloration can be altered by the number of melanocytes or the amount of pigment produced and stored in the organelles called MELANOSOMES. The large non-mammalian melanin-containing cells are called MELANOPHORES.
Nystagmus, Congenital
Nystagmus present at birth or caused by lesions sustained in utero or at the time of birth. It is usually pendular, and is associated with ALBINISM and conditions characterized by early loss of central vision. Inheritance patterns may be X-linked, autosomal dominant, or recessive. (Adams et al., Principles of Neurology, 6th ed, p275)
Pigmentation
Coloration or discoloration of a part by a pigment.
Albinism
General term for a number of inherited defects of amino acid metabolism in which there is a deficiency or absence of pigment in the eyes, skin, or hair.
SOXE Transcription Factors
A subclass of closely-related SOX transcription factors. Members of this subfamily have been implicated in regulating the differentiation of OLIGODENDROCYTES during neural crest formation and in CHONDROGENESIS.
Melanins
Insoluble polymers of TYROSINE derivatives found in and causing darkness in skin (SKIN PIGMENTATION), hair, and feathers providing protection against SUNBURN induced by SUNLIGHT. CAROTENES contribute yellow and red coloration.
Microphthalmia-Associated Transcription Factor
A basic helix-loop-helix leucine zipper transcription factor that regulates the CELL DIFFERENTIATION and development of a variety of cell types including MELANOCYTES; OSTEOCLASTS; and RETINAL PIGMENT EPITHELIUM. Mutations in MITF protein have been associated with OSTEOPETROSIS and WAARDENBURG SYNDROME.
Prader-Willi Syndrome
An autosomal dominant disorder caused by deletion of the proximal long arm of the paternal chromosome 15 (15q11-q13) or by inheritance of both of the pair of chromosomes 15 from the mother (UNIPARENTAL DISOMY) which are imprinted (GENETIC IMPRINTING) and hence silenced. Clinical manifestations include MENTAL RETARDATION; MUSCULAR HYPOTONIA; HYPERPHAGIA; OBESITY; short stature; HYPOGONADISM; STRABISMUS; and HYPERSOMNOLENCE. (Menkes, Textbook of Child Neurology, 5th ed, p229)
Chromosomes, Human, Pair 15
A specific pair of GROUP D CHROMOSOMES of the human chromosome classification.
Pigment Epithelium of Eye
The layer of pigment-containing epithelial cells in the RETINA; the CILIARY BODY; and the IRIS in the eye.
Hair
A filament-like structure consisting of a shaft which projects to the surface of the SKIN from a root which is softer than the shaft and lodges in the cavity of a HAIR FOLLICLE. It is found on most surfaces of the body.
Pedigree
The record of descent or ancestry, particularly of a particular condition or trait, indicating individual family members, their relationships, and their status with respect to the trait or condition.
Syndrome
A characteristic symptom complex.
Skin Diseases
Genome-Wide Association Study
An analysis comparing the allele frequencies of all available (or a whole GENOME representative set of) polymorphic markers in unrelated patients with a specific symptom or disease condition, and those of healthy controls to identify markers associated with a specific disease or condition.
Polymorphism, Single Nucleotide
A single nucleotide variation in a genetic sequence that occurs at appreciable frequency in the population.
Cattle
Domesticated bovine animals of the genus Bos, usually kept on a farm or ranch and used for the production of meat or dairy products or for heavy labor.
Iridaceae
A monocot plant family of the Liliopsida class. It is classified by some in the Liliales order and some in the Asparagales order.
Chediak-Higashi Syndrome
A form of phagocyte bactericidal dysfunction characterized by unusual oculocutaneous albinism, high incidence of lymphoreticular neoplasms, and recurrent pyogenic infections. In many cell types, abnormal lysosomes are present leading to defective pigment distribution and abnormal neutrophil functions. The disease is transmitted by autosomal recessive inheritance and a similar disorder occurs in the beige mouse, the Aleutian mink, and albino Hereford cattle.
Leishmania donovani
A parasitic hemoflagellate of the subgenus Leishmania leishmania that infects man and animals and causes visceral leishmaniasis (LEISHMANIASIS, VISCERAL). The sandfly genera Phlebotomus and Lutzomyia are the vectors.
Leishmaniasis, Visceral
A chronic disease caused by LEISHMANIA DONOVANI and transmitted by the bite of several sandflies of the genera Phlebotomus and Lutzomyia. It is commonly characterized by fever, chills, vomiting, anemia, hepatosplenomegaly, leukopenia, hypergammaglobulinemia, emaciation, and an earth-gray color of the skin. The disease is classified into three main types according to geographic distribution: Indian, Mediterranean (or infantile), and African.
Leishmaniasis, Cutaneous
An endemic disease that is characterized by the development of single or multiple localized lesions on exposed areas of skin that typically ulcerate. The disease has been divided into Old and New World forms. Old World leishmaniasis is separated into three distinct types according to epidemiology and clinical manifestations and is caused by species of the L. tropica and L. aethiopica complexes as well as by species of the L. major genus. New World leishmaniasis, also called American leishmaniasis, occurs in South and Central America and is caused by species of the L. mexicana or L. braziliensis complexes.
India
Phlebotomus
A genus of PSYCHODIDAE which functions as the vector of a number of pathogenic organisms, including LEISHMANIA DONOVANI; LEISHMANIA TROPICA; Bartonella bacilliformis, and the Pappataci fever virus (SANDFLY FEVER NAPLES VIRUS).
Antiprotozoal Agents
Substances that are destructive to protozoans.
Antimony Sodium Gluconate
Antimony complex where the metal may exist in either the pentavalent or trivalent states. The pentavalent gluconate is used in leishmaniasis. The trivalent gluconate is most frequently used in schistosomiasis.

The Sox10(Dom) mouse: modeling the genetic variation of Waardenburg-Shah (WS4) syndrome. (1/94)

Hirschsprung disease (HSCR) is a multigenic neurocristopathy clinically recognized by aganglionosis of the distal gastrointestinal tract. Patients presenting with aganglionosis in association with hypopigmentation are classified as Waardenburg syndrome type 4 (Waardenburg-Shah, WS4). Variability in the disease phenotype of WS4 patients with equivalent mutations suggests the influence of genetic modifier loci in this disorder. Sox10(Dom)/+ mice exhibit variability of aganglionosis and hypopigmentation influenced by genetic background similar to that observed in WS4 patients. We have constructed Sox10(Dom)/+ congenic lines to segregate loci that modify the neural crest defects in these mice. Consistent with previous studies, increased lethality of Sox10(Dom)/+ animals resulted from a C57BL/6J locus(i). However, we also observed an increase in hypopigmentation in conjunction with a C3HeB/FeJLe-a/a locus(i). Linkage analysis localized a hypopigmentation modifier of the Dom phenotype to mouse chromosome 10 in close proximity to a previously reported modifier of hypopigmentation for the endothelin receptor B mouse model of WS4. To evaluate further the role of SOX10 in development and disease, we have performed comparative genomic analyses. An essential role for this gene in neural crest development is supported by zoo blot hybridizations that reveal extensive conservation throughout vertebrate evolution and by similar Northern blot expression profiles between mouse and man. Comparative sequence analysis of the mouse and human SOX10 gene have defined the exon-intron boundaries of SOX10 and facilitated mutation analysis leading to the identification of two new SOX10 mutations in individuals with WS4. Structural analysis of the HMG DNA-binding domain was performed to evaluate the effect of human mutations in this region.  (+info)

Independent evaluation of onchocerciasis rapid assessment methods in Benue State, Nigeria. (2/94)

OBJECTIVE: To evaluate the prevalence of palpable nodules or skin depigmentation as rapid indicators of onchocerciasis epidemicity in at-risk communities. METHOD: We examined data collected in Benue State on 11035 individuals in 32 villages to evaluate these rapid assessment methods. RESULTS: The prevalence of palpable nodules correlates more closely with microfilarial prevalence (r=0.68, P<0.001) and community microfilarial load (r=0.64, P<0.001) than the prevalences of skin depigmentation or other potential rapid indicators. The recommended cut-off value for palpable nodules of 20% or more in males aged >20 years had a sensitivity of 94% and specificity of 50% compared to a cut-off of 40% or more for microfilarial prevalence in all ages. This would mean that in these 32 villages 17 of 18 would have been correctly identified for treatment, and a further 7 at lesser risk would have been targeted for treatment. CONCLUSIONS: Skin snipping and parasitological examination can be replaced by the simpler method of palpating onchocercal nodules to identify communities at serious risk of onchocerciasis. This has important operational benefits for onchocerciasis control programmes.  (+info)

A molecular analysis of the yemenite deaf-blind hypopigmentation syndrome: SOX10 dysfunction causes different neurocristopathies. (3/94)

The Yemenite deaf-blind hypopigmentation syndrome was first observed in a Yemenite sister and brother showing cutaneous hypopigmented and hyperpigmented spots and patches, microcornea, coloboma and severe hearing loss. A second case, observed in a girl with similar skin symptoms and hearing loss but without microcornea or coloboma, was reported as a mild form of this syndrome. Here we show that a SOX10 missense mutation is responsible for the mild form, resulting in a loss of DNA binding of this transcription factor. In contrast, no SOX10 alteration could be found in the other, severe case of the Yemenite deaf-blind hypopigmentation syndrome. Based on genetic, clinical, molecular and functional data, we suggest that these two cases represent two different syndromes. Moreover, as mutations of the SOX10 transcription factor were previously described in Waardenburg-Hirschsprung disease, these results show that SOX10 mutations cause various types of neurocristopathy.  (+info)

Successful bone marrow transplantation in a case of Griscelli disease which presented in accelerated phase with neurological involvement. (4/94)

Griscelli disease (GD) is a rare disorder characterized by pigment dilution, immunodeficiency and occurrence of accelerated phase consisting of hemophagocytosis, pancytopenia and neurological manifestations. Allogeneic BMT in the early period is an important modality of treatment for GD. We carried out an alloBMT from an HLA-identical sibling donor on a 4-year-old girl who presented in accelerated phase with neurological manifestations including convulsions, strabismus, severe dysarthria, ataxia and clonus. She was treated with etoposide, methylprednisolone and intrathecal methotrexate for 8 weeks and underwent alloBMT after receiving a conditioning regimen including ATG (rabbit, 10 mg/kg x 5 days), Bu/Cy. 8 x 108/kg nucleated bone marrow cells were given. Engraftment occurred early and the post-BMT period was uneventful. Currently, she is at 18 months post BMT with sustained engraftment and with a normal neurological examination except for minimal clonus. Long-term follow-up will determine the prognosis regarding the neurological findings.  (+info)

Spatially restricted hypopigmentation associated with an Ednrbs-modifying locus on mouse chromosome 10. (5/94)

We have used the varied expressivity of white spotting (hypopigmentation) observed in intrasubspecific crosses of Ednrb(s) mice (Mayer Ednrb(s)/Ednrb(s) and C3HeB/FeJ Ednrb(s)/Ednrb(s)) to analyze the effects of modifier loci on the patterning of hypopigmentation. We have confirmed that an Ednrb(s) modifier locus is present on mouse Chromosome 10. This locus is now termed k10, using the nomenclature established by Dunn in 1920. The k10(Mayer) allele is a recessive modifier that accounts for almost all of the genetic variance of dorsal hypopigmentation. Using intercross analyses we identified a second allele of this locus or a closely linked gene termed k10(C3H). The k10(C3H) allele is semidominant and is associated with the penetrance and expressivity of a white forelock phenotype similar to that seen in Waardenburg syndrome. Molecular linkage analysis was used to determine that the k10 critical interval was flanked by D10Mit10 and D10Mit162/D10Mit122 and cosegregates with mast cell growth factor (Mgf). Complementation crosses with a Mgf(Sl) allele (a 3-5-cM deletion) confirm the semidominant white forelock feature of the k10(C3H) allele and the dorsal spotting feature of K10(Mayer) allele. MgF was assessed as a candidate gene for k10(Mayer) and k10(C3H) by sequence and genomic analyses. No molecular differences were observed between the Mayer and C57BL/6J alleles of MgF; however, extensive genomic differences were observed between the C3HeB/FeJ and C57BL/6J alleles. This suggests that alteration of MgF expression in C3H mice may account for the k10(C3H) action on white forelock hypopigmentation. Crosses of Ednrb(s) with Kit(WJ-2) (the receptor for MGF)-deficient mice confirmed the hypothesis that synergistic interaction between the Endothelin and MGF signaling pathways regulates proper neural crest-derived melanocyte development in vivo.  (+info)

Tietz syndrome (hypopigmentation/deafness) caused by mutation of MITF. (6/94)

Patients with Tietz syndrome have congenital profound deafness and generalised hypopigmentation, inherited in a fully penetrant autosomal dominant fashion. The pigmentary features and complete penetrance make this syndrome distinct among syndromes with pigmentary anomalies and deafness, which characteristically have patchy depigmentation and variable penetrance. Only one family has been reported with the exact features described in the original report of this syndrome. This family was reascertained and a missense mutation was found in the basic region of the MITF gene in family members with Tietz syndrome. Mutations in other regions of this gene have been found to produce Waardenburg syndrome type 2 (WS2), which also includes pigmentary changes and hearing loss, but in contrast to Tietz syndrome, depigmentation is patchy and hearing loss is variable in WS2.  (+info)

Oa1 knock-out: new insights on the pathogenesis of ocular albinism type 1. (7/94)

Ocular albinism type I (OA1) is an X-linked disorder characterized by severe reduction of visual acuity, strabismus, photophobia and nystagmus. Ophthalmologic examination reveals hypopigmentation of the retina, foveal hypoplasia and iris translucency. Microscopic examination of both retinal pigment epithelium (RPE) and skin melanocytes shows the presence of large pigment granules called giant melanosomes or macromelanosomes. In this study, we have generated and characterized Oa1-deficient mice by gene targeting (KO). The KO males are viable, fertile and phenotypically indistinguishable from the wild-type littermates. Ophthalmologic examination shows hypopigmentation of the ocular fundus in mutant animals compared with wild-type. Analysis of the retinofugal pathway reveals a reduction in the size of the uncrossed pathway, demonstrating a misrouting of the optic fibres at the chiasm, as observed in OA1 patients. Microscopic examination of the RPE shows the presence of giant melanosomes comparable with those described in OA1 patients. Ultrastructural analysis of the RPE cells, suggests that the giant melanosomes may form by abnormal growth of single melanosomes, rather than the fusion of several, shedding light on the pathogenesis of ocular albinism.  (+info)

Ophthalmic manifestations of tuberous sclerosis: a population based study. (8/94)

BACKGROUND/AIMS: Tuberous sclerosis complex (TSC) has retinal and non-retinal ophthalmic manifestations. This study was designed to determine the prevalence of the ophthalmic manifestations and of refractive errors in a population of patients with TSC. METHODS: 179 patients identified were in a prevalence study of TSC in the south of England and 107 of these agreed to full ophthalmic examination which was successful in 100. Ophthalmic examination included examination of the eyelids, cover test, examination of the irides, dilation funduscopy using both direct and indirect ophthalmoscopy, and refraction using retinoscopy. Myopia was defined as a spherical equivalent <-0.5D and hyperopia as a spherical equivalent >+0.5D. RESULTS: Retinal hamartomas were seen in 44 of the 100 patients. The commonest morphological type of hamartoma seen was the flat, translucent lesion in 31 of the 44 patients (70%). The multinodular "mulberry" lesion was seen in 24 of the 44 patients (55%) and the transitional type lesion was seen in four of the 44 patients (9%). Punched out areas of retinal depigmentation were seen in 39 of the 100 patients but only six of 100 controls. 27% of eyes were myopic, 22% were hyperopic, and 27% had astigmatism >0.75D. Of the non-retinal findings, 39 patients had angiofibromas of the eyelids, five had non-paralytic strabismus, and three had colobomas. CONCLUSION: Apart from the higher prevalence of flat retinal hamartomas, the findings of this study compare closely with previous large clinic based series of TSC patients. Refractive findings were similar to previous studies of a similarly aged non-TSC population. This is the first series to document the statistically significant association of punched out chorioretinal depigmentation with TSC and the authors believe that it should be looked for as an aid to diagnosis.  (+info)

Osteoporosis and Oculocutaneous Hypopigmentation Syndrome disease: Malacards - Research Articles, Drugs, Genes, Clinical TrialsOsteoporosis and Oculocutaneous Hypopigmentation Syndrome disease: Malacards - Research Articles, Drugs, Genes, Clinical Trials
NIH Rare Diseases : 53 The following summary is from Orphanet, a European reference portal for information on rare diseases and orphan drugs.Orpha Number: 2786DefinitionOsteoporosis-oculocutaneous hypopigmentation syndrome is characterised by osteoporosis and congenital oculocutaneous hypopigmentation. Three cases have been described in the literature. The mode of inheritance appears to be autosomal recessive.Visit the Orphanet disease page for more resources ...
https://www.malacards.org/card/osteoporosis_and_oculocutaneous_hypopigmentation_syndrome
Oculocerebral hypopigmentation syndrome (Cross syndrome) in a gypsy child | Hereditary Ocular DiseasesOculocerebral hypopigmentation syndrome (Cross syndrome) in a gypsy child | Hereditary Ocular Diseases
Courtens W, Broeckx W, Ledoux M, Vamosa E. Oculocerebral hypopigmentation syndrome (Cross syndrome) in a Gipsy child. Acta Paediatr Scand. 1989 Sep;78(5):806-10.. ...
https://disorders.eyes.arizona.edu/references/oculocerebral-hypopigmentation-syndrome-cross-syndrome-gypsy-child
Hypopigmentation and Maternal-Zygotic Embryonic Lethality Caused by a Hypomorphic Mbtps1 Mutation in Mice<...
TY - JOUR. T1 - Hypopigmentation and Maternal-Zygotic Embryonic Lethality Caused by a Hypomorphic Mbtps1 Mutation in Mice. AU - Rutschmann, Sophie. AU - Crozat, Karine. AU - Li, Xiaohong. AU - Du, Xin. AU - Hanselman, Jeffrey C.. AU - Shigeoka, Alana A.. AU - Brandl, Katharina. AU - Popkin, Daniel L.. AU - McKay, Dianne B.. AU - Xia, Yu. AU - Moresco, Eva Marie Y. AU - Beutler, Bruce. PY - 2012/4. Y1 - 2012/4. N2 - The site 1 protease, encoded by Mbtps1, mediates the initial cleavage of site 2 protease substrates, including sterol regulatory element binding proteins and CREB/ATF transcription factors. We demonstrate that a hypomorphic mutation of Mbtps1 called woodrat (wrt) caused hypocholesterolemia, as well as progressive hypopigmentation of the coat, that appears to be mechanistically unrelated. Hypopigmentation was rescued by transgenic expression of wild-type Mbtps1, and reciprocal grafting studies showed that normal pigmentation depended upon both cell-intrinsic or paracrine factors, as ...
https://utsouthwestern.pure.elsevier.com/en/publications/hypopigmentation-and-maternal-zygotic-embryonic-lethality-caused-
Albinism | Hypopigmentation - Types Symptoms Diagnosis Treatment FAQsAlbinism | Hypopigmentation - Types Symptoms Diagnosis Treatment FAQs
Albinism or hypopigmentation is a genetic disorder characterized by the complete or partial absence of pigment called melanin in the skin, hair and eyes. There is no known way to prevent albinism or hypopigmentation.
http://www.medindia.net/patients/patientinfo/Albinism.htm
Common Causes and Treatments for Hypopigmentation: White Spots on Skin
When skin is damaged by UV exposure two conditions can occur: hyperpigmentation, and hypopigmentation. The first, hyperpigmentation, is the occurance of dark
http://diyhomelivingstyle.com/common-causes-and-treatments-for-hypopigmentation-white-spots-on-skin/
Hypopigmentation Archives - Cosmedic ClinicHypopigmentation Archives - Cosmedic Clinic
Hypopigmentation is the loss of skin colour. It is caused by melanocyte or melanin depletion, or a decrease in the amino acid tyrosine, which is used by melanocytes to make melanin.. ...
https://www.cosmedic-clinic.co.uk/shop/indication/hypopigmentation/?filter_indication=anti-ageing&query_type_indication=or
Hypopigmentation | Ag3dermHypopigmentation | Ag3derm
Hypopigmentation You must have heard a lot about pigmentation and hyper pigmentation. But it is possible that you do not know anything much about
http://www.ag3derm.com/hypopigmentation/
Oculocerebral syndrome with hypopigmentation Symptoms, Diagnosis, Treatments and Causes - RightDiagnosis.comOculocerebral syndrome with hypopigmentation Symptoms, Diagnosis, Treatments and Causes - RightDiagnosis.com
Oculocerebral syndrome with hypopigmentation information including symptoms, diagnosis, misdiagnosis, treatment, causes, patient stories, videos, forums, prevention, and prognosis.
http://www.rightdiagnosis.com/o/oculocerebral_syndrome_with_hypopigmentation/intro.htm
Hypopigmentation Causes: White Spots & Patches On The SkinHypopigmentation Causes: White Spots & Patches On The Skin
Whilst hypopigmentation simply means a lack of pigment in the skin, there can be a number of different causes. Here we briefly cover each of those different conditions.
https://www.smacr.com/hypopigmentation/causes/
A Review of Skin Hypopigmentation and Contemporary Strategies to Achieve an Even Skin ToneA Review of Skin Hypopigmentation and Contemporary Strategies to Achieve an Even Skin Tone
Whether formulating for Asia, North America, Europe or Latin America, the cosmetic chemist will face one common demand in all these markets: an even skin tone. This article provides an overview of chemical and biological agents capable of causing hypopigmentation via interactions through different stages of the melanogenic pathway.
http://www.cosmeticsandtoiletries.com/research/biology/premium-A-Review-of-Skin-Hypopigmentation-and-Contemporary-Strategies-to-Achieve-an-Even-Skin-Tone-223359081.html
Suffering from chronic Idiopathic Guttate Hypomelanosis skin condition. Cost of treatment, healing time? - Doctors insight on...Suffering from chronic Idiopathic Guttate Hypomelanosis skin condition. Cost of treatment, healing time? - Doctors insight on...
Question - Suffering from chronic Idiopathic Guttate Hypomelanosis skin condition. Cost of treatment, healing time?. Ask a Doctor about diagnosis, treatment and medication for Idiopathic guttate hypomelanosis, Ask a Dermatologist
http://www.healthcaremagic.com/questions/Suffering-from-chronic-Idiopathic-Guttate-Hypomelanosis-skin-condition-Cost-of-treatment-healing-time/312931
Leukonychia Punctata: Causes & Diagnoses | Symptoma.comLeukonychia Punctata: Causes & Diagnoses | Symptoma.com
Leukonychia Punctata Possible Causes (Differential Diagnoses) include ❗ Congenital Leukonychia ❗ Rheumatoid Arthritis ❗ Diabetes Mellitus ❗ Check more at Symptoma.com
https://www.symptoma.com/en/ddx/leukonychia-punctata
Remove leukonychia - New Doctor InsightsRemove leukonychia - New Doctor Insights
Answers from trusted physicians on remove leukonychia. First: excellent and detailed description: http://www.aafp.org/afp/2002/0615/p2547.html
https://www.healthtap.com/topics/remove-leukonychia
NOAH Ed U | National Organization for Albinism and HypopigmentationNOAH Ed U | National Organization for Albinism and Hypopigmentation
NOAH presents this complimentary supplement, geared toward educators, based on its quarterly magazine, Albinism InSight. Sign up to receive the to be placed on the distribution list.
https://www.albinism.org/ed-u/
NOAH SchoolKit | National Organization for Albinism and HypopigmentationNOAH SchoolKit | National Organization for Albinism and Hypopigmentation
The SchoolKit provides information and resources to guide parents in supporting teachers when a student with albinism will be in their classroom.
https://albinism.org/schoolkit/
IDEALDERM ® Viti Capsules for skin hypopigmentation
Its innovative complex of active ingredients contributes to: 1) the normal skin pigmentation (face and body), 2) the protection of cells from oxidative stress, 3) the normal function of the immune system.
https://www.vitiligo.de/contents/en-uk/p1001_buy_vitiligo_gel_cream_capsules.html
Zevelekakis. Hypopigmentation
There are many disorders of a lack of pigmentation in the skin, with different causations and effects, of which vitiligo is only the best known; this comprehensive text from international experts will enable clinicians to diagnose the full range of these conditions and suggest the most effective management options for their patients.
http://site.zevelekakis.gr/hypopigmentation
How Do You Treat White Spots on Skin?How Do You Treat White Spots on Skin?
Hypopigmentation is a condition in which the skin is lighter in color than normal. Treating hypopigmentation may involve the use of topical corticosteroids or tars, light or laser treatment, or surgical skin grafting.
https://www.rxlist.com/how_do_you_treat_white_spots_on_skin/article.htm
The use of ginger to treat skin hypopigmentation:: I cant believe I found the solutionThe use of ginger to treat skin hypopigmentation:: I cant believe I found the solution
Seeing myself in the mirror was unbearable. The contrast of the all-white patches was obvious and VERY noticeable. I cried for months and refused to go to school or even see my friends. I couldnt look at people in the eyes and much less get a boyfriend. No make-up could hide it, and I was too young and too naive to know how to apply it. I lost many precious moments of my teenage years to the condition ...
http://gingerhypopigmentation.blogspot.com/2008/10/i-cant-believe-i-found-cure.html?showComment=1475834348137
HYPOPIGMENTATION | SelfDecode | Genome AnalysisHYPOPIGMENTATION | SelfDecode | Genome Analysis
Selected genes are highlighted in orange, bookmarked genes are green. - Chemical increases gene, - Chemical decreases gene, - Chemical increases and decreases gene simultaneosly, No arrows - gene doesnt interact with the chemical. - Gene should be increased/decreased most of the time and the chemical does it. - Gene should be increased/decreased most of the time but the chemical does the opposite. ...
https://selfdecode.com/disease/hypopigmentation/
Melanoma-associated hypopigmentation as sign of EMT-propelled metastatic spread: towards identification of the molecular...Melanoma-associated hypopigmentation as sign of EMT-propelled metastatic spread: towards identification of the molecular...
We use cookies to ensure that we give you the best experience on our website. If you continue to use this site we will assume that you agree with it.I AgreeData Privacy Policy ...
https://www.iegt-rostock.de/en/?abstracts=melanoma-associated-hypopigmentation-sign-emt-propelled-metastatic-spread-towards-identification-molecular-particularities-2
Hypopigmentation | DermpediaHypopigmentation | Dermpedia
The first contribution to the chapter will require editorial approval before being published. Only high quality extensive reviews will be approved for initial publication.. Publishing on Dermpedia is easy. If you already have suitable content on file, it will not take much more than cut and paste to publish it on Dermpedia. Please see help for instructions. Dermpedia is designed to be user-editable, but yes, if you have great content, would like to become the Editor of this page, but have not time to edit this page, we will find helpers to publish it for you.. Registration is required.. Adding Cases and Images. You can add cases to book pages and either add images directly or attached to cases (preferred).. Add a case: Click on the Add a case link while on the book page and you will be asked to create a new case by filling in the necessary fields and pressing submit. Once youve done that, there is the further opportunity to add images to the case.. Add an image: Click on the Add an image ...
http://www.dermpedia.org/dermpedia-textbook/hypopigmentation
Prolonged teats generic viagra echocardiogram imminent recovering hypopigmentation, ventricle. - IELTS Band
The |a href=http://buy-retin-aonline.net/#retin-a-www.ieltsband.com|retin a tretinoin ... /a| nodules pairs cricoid verbalizing, going, teenagers.
http://ieltsband.net/index.php?qa=1049&qa_1=prolonged-echocardiogram-recovering-hypopigmentation-ventricle
Morphologic Changes in Retinal Pigment Epithelium (RPE) of the Hypopigmented Zebrafish Mutant, Golden (golb1) | IOVS | ARVO...Morphologic Changes in Retinal Pigment Epithelium (RPE) of the Hypopigmented Zebrafish Mutant, Golden (golb1) | IOVS | ARVO...
Purpose:: The zebrafish mutant, golden, is hypopigmented due to a mutation in a novel cation exchanger, NCKX5 (SLC24A5). Pigmentation has been shown, at least epidemiologically, to be an important factor in age-related macular degeneration (AMD). In addition, NCKX5 has been demonstrated in human RPE, and the gene encoding NCKX5 is located on chromosome 15q21, a region that also has a major susceptibility locus associated with AMD. The aim of this study is to evaluate the morphologic features of the retina/RPE of the golden zebrafish in comparison with human AMD in order to establish a possible animal model for this disease. Methods:: Wildtype (Lyles) and mutant (golden, golb1) zebrafish (sacrificed in light) were fixed in paraformaldehyde or glutaraldehyde. Eyes were removed and investigated by light (LM) or electron microscopy (EM), respectively. 22 eyes were obtained from wildtype (wt), age range 8-34 months, and 18 from golden, age range 9-33 months. Results:: By LM, there was an obvious ...
https://iovs.arvojournals.org/article.aspx?articleid=2383493
Hypomelanosis of ItoHypomelanosis of Ito
Psychiatry healthcare professionals gain a thorough knowledge base of psychiatric disorder information to offer the best patient care. Get our FREE app now.
http://www.psychiatryadvisor.com/pediatrics/hypomelanosis-of-ito/article/624117/
How common are albino children? | Reference.comHow common are albino children? | Reference.com
According to the National Organization for Albinism and Hypopigmentation, only one out of 17,000 children has some form of albinism. Albinism is caused by a severe lack of the pigment...
https://www.reference.com/family/common-albino-children-df505dd3a578b85b
OMIA - Online Mendelian Inheritance in AnimalsOMIA - Online Mendelian Inheritance in Animals
Mendelian trait/disorder: yes Mode of inheritance: Autosomal Recessive Considered a defect: no Key variant known: yes Year key variant first reported: 2015 Key variant is published: no Species-specific symbol: OH History: As reported by Hollman et al. (2017), via a personal communication from Dr Jon Beever, Recently an alteration of iris coloration has been observed in Angus and Simmental breed. Affected cattle showed an oculocutaneous hypopigmentation (OH) with a pale blue iris and a tan periphery coupled with a change in coat color from black to chocolate. It is assumed that this aberration was introduced into the Simmental breed in the late 1950s by Angus founders and is inherited as an autosomal recessive trait. As described by Dr Beever in a 2015 press release from the American Simmental Association (Anon., 2015), In the spring of 2012, the American Simmental Association (ASA) received an abnormality report indicating the occurrence of a newborn calf with white-colored eyes and a ...
https://omia.org/OMIA002101/9913/
Her Little EyeHer Little Eye
Ive created Carrahs Bows to do just that. I own an online boutique called PrissyMiss Boutique where I make and sell HairBows among other Prissy things. I figured since I was already making beautiful HairBows, why not do it for a great cause that is very close to my heart. I have promised to donate $2 from each sale of a Carrahs Bow to NOAH (The National Organization for Albinsim and Hypopigmentation) in hope of letting more people know the truth about all of the beautiful people with Albinism ...
http://faircarebear.blogspot.com/2009/06/her-little-eye.html
Developmental MilestonesDevelopmental Milestones
Ive created Carrahs Bows to do just that. I own an online boutique called PrissyMiss Boutique where I make and sell HairBows among other Prissy things. I figured since I was already making beautiful HairBows, why not do it for a great cause that is very close to my heart. I have promised to donate $2 from each sale of a Carrahs Bow to NOAH (The National Organization for Albinsim and Hypopigmentation) in hope of letting more people know the truth about all of the beautiful people with Albinism ...
http://faircarebear.blogspot.com/2009/05/developmental-milestones.html
Histologic analysis of photochemical lesions produced in rhesus retina by short-wave-length light. | IOVS | ARVO JournalsHistologic analysis of photochemical lesions produced in rhesus retina by short-wave-length light. | IOVS | ARVO Journals
The photopathology of retinal lesions produced by extended exposure (1000 sec) to low corneal power levels (62 microW) of blue light (441 nm) was investigated by light microscopy in 20 rhesus eyes over an interval ranging from 1 hr to 90 days after exposure. Results indicate a nonthermal type of photochemical lesion originating in the retinal pigment epithelium and leading to a histological response with hypopigmentation which requires 48 hr to appear. This type of lesion helps to explain solar retinitis and eclipse blindness and has significance for aging and degenerative changes in the retina.. ...
http://iovs.arvojournals.org/article.aspx?articleid=2175470
Semi Permanent Make up | CC Kat Aesthetics, BirminghamSemi Permanent Make up | CC Kat Aesthetics, Birmingham
Permanent cosmetics is for people wanting enhancements to their make-up, hypopigmentation from scarring and nipple areola pigmentation post-reconstruction surgery.
http://cckat.com/procedures/semi-permanent-makeup?p=2
The Truth About Skin Color Side Effects After Laser TreatmentThe Truth About Skin Color Side Effects After Laser Treatment
How common is hypopigmentation after Q-switch 1064nm lasers? Without seeing the full picture, many people who have undergone laser treatments are succumbing to unnecessary panic due to misinformation about what lasers can and cannot do for their skins. Read more: The Secret Facial Mist That Gives This Game of Thrones Star Glowy Skin MYTH:…
https://skinreporter.wordpress.com/2017/12/21/the-truth-about-skin-color-side-effects-after-laser-treatment/?like_comment=29&_wpnonce=f0b6817dcb
Usage of Eidel cream for skin rashes caused white patches. Will it likely fade?Usage of Eidel cream for skin rashes caused white patches. Will it likely fade?
Hypopigmentation caused by using certain drugs can fade away after stopping the usage for a while. Consult a specialist o know more.
https://www.icliniq.com/qa/hypopigmented-patches/will-eidel-induced-hypopigmented-patch-on-face-fade-on-its-own
TOP 5 PROCEDURES TO IMPROVE YOUR JAWLINE IN 2019 | The Aesthetic CliniqueTOP 5 PROCEDURES TO IMPROVE YOUR JAWLINE IN 2019 | The Aesthetic Clinique
It appears that 2019 is the Year of the Jawline. A strong jawline is interpreted as a characteristic of strength, beauty, virility, and power. In a study of S&P 500 CEOs, 95% were considered to have a well defined jawline. With advances in aesthetic technology and techniques, achieving that perfect profile is becoming easier, with less downtime, costs, and risks. Here are the top 5 procedures to improve your jawline:. 5) Neck Lift/Lower Face Lift: With incisions placed behind the ears, this surgery can be performed with just local anesthetic. Downtime is about a week or 2. For severe jowling and platysma bands, this will give the best results.. 4) Ablative Laser (Erbium/CO2) or Renuvion(J Plasma): These procedures have significant wound care for a week or more. The skin is stimulated by the heat of the laser/plasma to contract and create collagen. Healing time can take weeks and redness can persist for months. Risks include infection, scarring, hyper and hypopigmentation. Procedure is limited ...
https://theclinique.com/2019/01/29/top-5-procedures-to-improve-your-jawline-in-2019/
Participant 138Participant 138
Male, age 3 with developmental delay, abnormal muscle tone and movement, hypopigmentation, and a variant in the EZH1 gene. Looking for similar patients!
https://undiagnosed.hms.harvard.edu/participants/participant-138/
Dr Ho Rui Ming Doctor×DentistDr Ho Rui Ming Doctor×Dentist
Hi allswell,. It does look like abit of hypopigmentation and you are correct that it is difficult to treat (most of the time we will have to rely on our own cells to repigment the area unless you explore other […]. ...
http://doctorxdentist.com/members/rui-ming-ho/
Progressive macular hypomelanosis, excellent response with narrow-band ultraviolet B phototherapy - Montero - 2011 -...Progressive macular hypomelanosis, excellent response with narrow-band ultraviolet B phototherapy - Montero - 2011 -...
Progressive macular hypomelanosis (PMH) is an acquired disorder of skin pigmentation, which is mostly under-diagnosed. It is characterized by nummular hypopigmented lesions appearing on the trunk in young persons. Several treatment options are available, although topical clindamycin and benzoyl peroxide have been used traditionally. However, good results have recently been achieved using narrow-band ultraviolet B (NBUVB) phototherapy. We present the case of a 13-year-old girl with hypopigmented lesions on the trunk and limbs that had progressed over 1 year and that were diagnosed as PMH. The patient was initially treated with topical clindamycin and benzoyl peroxide. However, little improvement was seen and treatment was then started with NBUVB phototherapy. After 25 sessions, with a total cumulative dose of 18 J/cm2, the patient showed almost total repigmentation of the lesions. The treatment of PMH is often difficult, and very little is currently known about the treatment response in this ...
http://onlinelibrary.wiley.com/doi/10.1111/j.1600-0781.2011.00583.x/full
Skin hypopigmentation | definition of skin hypopigmentation by Medical dictionarySkin hypopigmentation | definition of skin hypopigmentation by Medical dictionary
Looking for online definition of skin hypopigmentation in the Medical Dictionary? skin hypopigmentation explanation free. What is skin hypopigmentation? Meaning of skin hypopigmentation medical term. What does skin hypopigmentation mean?
https://medical-dictionary.thefreedictionary.com/skin+hypopigmentation
Hypomelanosis of Ito - American Osteopathic College of Dermatology (AOCD)
The skin findings associated with hypomelanosis of Ito include small patches of pale or white skin that merge to form larger, asymmetric patches arranged in whorled or linear patterns. These patterns follow the lines of Blaschko which represent a developmental pattern of the skin. The pale or hypopigmented areas can be found in any part of the body including the head, face, neck, trunk and extremities. Typically a v-shape pattern is seen over the face and spine, an s-shape pattern is seen over the trunk, and a linear pattern is seen over the extremities. Additionally, it is important to note that the hypopigmentation is not preceded by vesicular or verrucous stages as this is a hallmark of another disorder that appears similar to hypomelanosis of Ito, known as incontinentia pigmenti.. Hair abnormalities can also be seen with hypomelanosis of Ito and can include changes in hair color, hair breakage and/or loss as well as hair with a white-gray color.. Individuals with hypomelanosis of Ito may ...
http://www.aocd.org/?page=HypomelanosisofIto
Acne-like leg skin symptoms and Chronic acne-like cheek symptoms and Hypopigmentation in children and Hypopigmented lesions in...Acne-like leg skin symptoms and Chronic acne-like cheek symptoms and Hypopigmentation in children and Hypopigmented lesions in...
List of 35 causes for Acne-like leg skin symptoms and Chronic acne-like cheek symptoms and Hypopigmentation in children and Hypopigmented lesions in children, alternative diagnoses, rare causes, misdiagnoses, patient stories, and much more.
https://symptoms.rightdiagnosis.com/cosymptoms/acne-like-leg-skin-symptoms/chronic-acne-like-cheek-symptoms/hypopigmentation-in-children/hypopigmented-lesions-in-children.htm
Acquired leukoderma | definition of acquired leukoderma by Medical dictionaryAcquired leukoderma | definition of acquired leukoderma by Medical dictionary
Looking for online definition of acquired leukoderma in the Medical Dictionary? acquired leukoderma explanation free. What is acquired leukoderma? Meaning of acquired leukoderma medical term. What does acquired leukoderma mean?
https://medical-dictionary.thefreedictionary.com/acquired+leukoderma
A genome-wide association study reveals a locus for bilateral iridal hypopigmentation in Holstein Friesian cattle | BMC Genomic...A genome-wide association study reveals a locus for bilateral iridal hypopigmentation in Holstein Friesian cattle | BMC Genomic...
Eye pigmentation abnormalities in cattle are often related to albinism, Chediak-Higashi or Tietz like syndrome. However, mutations only affecting pigmentation of coat color and eye have also been described. Herein 18 Holstein Friesian cattle affected by bicolored and hypopigmented irises have been investigated. Affected animals did not reveal any ophthalmological or neurological abnormalities besides the specific iris color differences. Coat color of affected cattle did not differ from controls. Histological examination revealed a reduction of melanin pigment in the iridal anterior border layer and stroma in cases as cause of iris hypopigmentation. To analyze the genetics of the iris pigmentation differences, a genome-wide association study was performed using Illumina BovineSNP50 BeadChip genotypes of the 18 cases and 172 randomly chosen control animals. A significant association on bovine chromosome 8 (BTA8) was identified at position 60,990,733 with a -log10(p) = 9.17. Analysis of genotypic and
https://bmcgenomdata.biomedcentral.com/articles/10.1186/s12863-017-0496-4
What is your diagnosis? : Journal of the Egyptian Womens Dermatologic SocietyWhat is your diagnosis? : Journal of the Egyptian Womens Dermatologic Society
White nails or leukonychia is the most common chromatic abnormality of the nails 1. Leukonychia may be acquired or inherited and can be divided into (a) true leukonychia due to matrix involvement, (b) apparent leukonychia due to subungual tissue abnormality, and (c) pseudoleukonychia due to external factors affecting the nail plate 1.. Acquired leukonychia may be caused by a wide range of abnormalities; however, it is frequently associated with trauma (most common), drugs such as chemotherapeutic agents 2, infections, liver cirrhosis, ulcerative colitis or leprosy, hypocalcaemia and various dermatoses among others 1.. The clinical presentation of leukonychia ranges from complete nail whitening to discrete white punctata or striae 3. True leukonychia punctata is the most common form of leukonychia, in which small white spots appear on the nails as a result of repeated minor trauma to the matrix, such as picking and biting of the nails in young children and nail biters 1,4.. ...
https://journals.lww.com/jewds/fulltext/2018/09000/what_is_your_diagnosis_.11.aspx
Leukonychia - WikipediaLeukonychia - Wikipedia
Leukonychia (or leuconychia), also known as white nails or milk spots, is a medical term for white discolouration appearing on nails. It is derived from the Greek words leuko (white) and nychia (nails). The most common cause is injury to the base of the nail (the matrix) where the nail is formed. It is harmless and most commonly caused by minor injuries, such as nail biting, that occur while the nail is growing. Leukonychia occurs most commonly in healthy individuals, unrelated to any known nutritional or physiological deficiency. When caused by injury the marks will disappear as the nail grows outwards, however a dietary deficiency will cause recurrent leukonychia. Other possible reasons for this problem with nail colour can be linked to: Arsenic poisoning Lead poisoning Pneumonia Heart disease Renal failure Ill health Hypoalbuminemia Vitamin deficiency Ulcerative colitis Hepatic cirrhosis Psychogenic stresses Onychophagia Occupational trauma Zinc deficiency Protein deficiency Psoriasis as ...
https://en.wikipedia.org/wiki/Leukonychia
Acne-like leg skin symptoms and Chronic acne-like cheek symptoms and Flat white skin lesions in children and Hypopigmentation...Acne-like leg skin symptoms and Chronic acne-like cheek symptoms and Flat white skin lesions in children and Hypopigmentation...
List of 30 causes for Acne-like leg skin symptoms and Chronic acne-like cheek symptoms and Flat white skin lesions in children and Hypopigmentation in children, alternative diagnoses, rare causes, misdiagnoses, patient stories, and much more.
https://symptoms.rightdiagnosis.com/cosymptoms/acne-like-leg-skin-symptoms/chronic-acne-like-cheek-symptoms/flat-white-skin-lesions-in-children/hypopigmentation-in-children.htm
The use of ginger to treat skin hypopigmentation:: The use of fresh ginger root to treat and reverse skin hypopigmentation and...The use of ginger to treat skin hypopigmentation:: The use of fresh ginger root to treat and reverse skin hypopigmentation and...
I was sitting at my orthodontists chair when my orthodontists assistant, who was doing some work in my mouth, said: You have discoloration on your chin, I see. Here it comes again, I thought. One more comment about my hypo. As if I needed a reminder, I thought. Yep, I replied. Ive had it for 12 years. Wow, thats a long time, she said. My mother and I have it too, but we got it under control, she said. I immediately jumped out of the chair and took a look at her face. Not one sign of discoloration. HOW DID YOU GET RID OF IT? I asked. Well, she said, I was on treatment for years and years without success. The latest technology on the market is a cleaning solution that I was supposed to use to clean the area first. That solution cost me about $300. After I prepared the affected area with the solution, I was supposed to put on a cream which cost me about $1,000 a tube. Im not using that anymore. LET ME BUY IT FROM YOU! I exclaimed. I had gone to the dermatologist a ...
http://gingerhypopigmentation.blogspot.com/2008/10/use-of-fresh-ginger-root-to-treat-and.html?showComment=1393096438169
Familial progressive cardiac conduction defect
Familial progressive cardiac conduction defect (PCCD) is a is a cardiac (heart) conduction disorder that may progress to complete heart block. Affected people may not have any symptoms, or the condition may cause shortness of breath, dizziness, fainting, abdominal pain, heart failure, or sudden death. Mutations in several genes, including the SCN5A, SCN1B and TRPM4 genes, can cause PCCD. Several other genes may be the cause when PCCD occurs with congenital heart disease. Familial PCCD is usually inherited in an autosomal dominant manner. However, not all people that have the mutated gene will have the condition; in those that do, symptoms and severity can vary (known as reduced penetrance and variable expressivity). Autosomal recessive inheritance and sporadic cases have been reported, but are rare. Treatment includes implantation of a pacemaker ...
http://diseaseinfosearch.org/Familial+progressive+cardiac+conduction+defect/9781
Familial progressive cardiac conduction defect | Genetic and Rare Diseases Information Center (GARD) - an NCATS...Familial progressive cardiac conduction defect | Genetic and Rare Diseases Information Center (GARD) - an NCATS...
A collection of disease information resources and questions answered by our Genetic and Rare Diseases Information Specialists for Familial progressive cardiac conduction defect
https://rarediseases.info.nih.gov/diseases/10005/familial-progressive-cardiac-conduction-defect
Leukoderma - Dr.Tatheds HomeopathyLeukoderma - Dr.Tatheds Homeopathy
Chinchwad:Opp. Lokmanya Hospital, Pune - 411 033 020-27455480Call us today! Opening Hours Mon - Sat: 10:00 - 19:00 Book Appointment Leukoderma Home / Leukoderma Treatment Leukoderma Vitiligo is a condition in which the skin turns white due to ,loss of pigment - melanin which is produced by melanocytes which gives colour to skin . In … Leukoderma Read More »
https://drtathed.com/treatments/leukoderma
JCI - Griscelli syndrome restricted to hypopigmentation results from a melanophilin defect (GS3) or a MYO5A F-exon deletion ...JCI - Griscelli syndrome restricted to hypopigmentation results from a melanophilin defect (GS3) or a MYO5A F-exon deletion ...
Griscelli syndrome (GS; MIM 214450) is a rare autosomal recessive disorder that results in a characteristic pigmentary dilution of the skin and the hair, with the presence of large clumps of pigment in hair shafts and an abnormal accumulation of end-stage melanosomes in the center of melanocytes (1-4). So far, two forms of GS have been described. Type 1 (GS1) associates characteristic albinism with a severe primary neurological impairment. Patients exhibit severe developmental delay and mental retardation occurring early in life. These patients carry mutations of the myosin 5A gene (MYO5A) (2), which encodes an organelle motor protein, Myosin Va (MyoVa), with a determining role in neuron function (5). The second type of Griscelli syndrome (GS2) is characterized by the same hypopigmentation associated with an immune defect, leading to episodes of a life-threatening uncontrolled T lymphocyte and macrophage activation syndrome known as accelerated phase or hemophagocytic syndrome (HS) (6). During ...
https://www.jci.org/articles/view/18264
Enophthalmos disease: Malacards - Research Articles, Drugs, Genes, Clinical TrialsEnophthalmos disease: Malacards - Research Articles, Drugs, Genes, Clinical Trials
MalaCards based summary : Enophthalmos is related to yemenite deaf-blind hypopigmentation syndrome and intraocular pressure quantitative trait locus. An important gene associated with Enophthalmos is ERCC6 (ERCC Excision Repair 6, Chromatin Remodeling Factor). The drugs Ketamine and Bupivacaine have been mentioned in the context of this disorder. Affiliated tissues include bone, eye and breast, and related phenotypes are nervous system and vision/eye ...
https://www.malacards.org/card/enophthalmos?search=fbn1
Are the white spots on my skin vitiligo or idiopathic guttate hypomelanosis?Are the white spots on my skin vitiligo or idiopathic guttate hypomelanosis?
I am a 50-year-old brown skinned man. During the preceding couple of months, 8-10 |b|white spots, 2-4 mm in diameter, have developed|/b| on my right shin, right thigh, left forearm, near left palm, left side of waist, and abdomen. In addition, some pale and many black/dark brown spots have also appeared on both my legs, thighs, forearms, waist and abdomen. Till now, the total number of light coloured/pale spots is 10-12 and that of black/dark brown is 15-20. There is no significant increase in the size or number of the white spots during the last one month. However, the number of light coloured and black spots has gone up a little. Almost all spots are smooth and not causing any discomfort. I have consulted two dermatologists. One says it is vitiligo. However, the other says it is idiopathic guttate hypomelanosis (IGH), a common problem of middle-aged people and has not recommended any treatment for it. I am totally confused and under severe psychological stress of vitiligo and dont understand what to
https://doctor.ndtv.com/faq/are-the-white-spots-on-my-skin-vitiligo-or-idiopathic-guttate-hypomelanosis-5046
Concealing Hypo & Hyperpigmentation with Foundation: The Techniques Ma - OxygenetixConcealing Hypo & Hyperpigmentation with Foundation: The Techniques Ma - Oxygenetix
Having skin discoloration such as hypopigmentation or hyperpigmentation can be life-altering. Hypopigmentation shows up as a loss of pigment in splotches on the skin, and on the other hand, hyperpigmentation comes forth as darker pigment than the natural skin color. Having abnormal pigmentation may affect your mental
https://www.oxygenetix.com/fr/blogs/blog/hypo-or-hyper-pigmentation-whats-the-difference-and-how-you-can-cover-it
Mycosis Fungoides Presenting Initially as Cutaneous Hypopigmentation | NTU ScholarsMycosis Fungoides Presenting Initially as Cutaneous Hypopigmentation | NTU Scholars
臺大位居世界頂尖大學之列，為永久珍藏及向國際展現本校豐碩的研究成果及學術能量，圖書館整合機構典藏（NTUR）與學術庫（AH）不同功能平台，成為臺大學術典藏NTU scholars。期能整合研究能量、促進交流合作、保存學術產出、推廣研究成果。. To permanently archive and promote researcher profiles and scholarly works, Library integrates the services of NTU Repository with Academic Hub to form NTU Scholars.. ...
http://scholars.lib.ntu.edu.tw/handle/123456789/327039
What Causes White Scars or Hypopigmentation - Hypophil
Statistics has shown that between 35 and 70 million people worldwide live with vitiligo.. Generally, signs of vitiligo begin to appear before the age of 20, but they can show up at any time. Beneath the skin, vitiligo can be associated with a greater risk of certain medical issues like celiac diseases, Type 1 diabetes and rheumatoid arthritis.. Causes of Vitiligo. The exact cause of vitiligo is still unknown. Scientists and doctors have theories about causes of this disorder. The most common form being an autoimmune disease, which means that a persons own immune system accidentally attacks the skin pigment cells also known as melanocytes. And as a result can no longer produce pigments in normal amount, which then causes the skin to turn white (de-pigmentation). Heredity may also be a factor as there are increased incidents of vitiligo in some families. Some believe single events such as sunburn or emotional distress triggers this condition. However, none of these theories has been proved to be ...
https://www.hypophil.com/what-causes-white-scars-and-hypopigmentation/
A Review of Skin Hypopigmentation and Contemporary Strategies to Achieve an Even Skin ToneA Review of Skin Hypopigmentation and Contemporary Strategies to Achieve an Even Skin Tone
Whether formulating for Asia, North America, Europe or Latin America, the cosmetic chemist will face one common demand in all these markets: an even skin tone. This article excerpt provides an overview of melanosome manipulation.
http://www.cosmeticsandtoiletries.com/formulating/category/skincare/173141691.html?page=2
METHOD AND APPARATUS FOR DERMATOLOGICAL HYPOPIGMENTATION - Patent applicationMETHOD AND APPARATUS FOR DERMATOLOGICAL HYPOPIGMENTATION - Patent application
0041] The protrusions 110 can be provided with an approximately circular cross-sectional shape. The cross-sectional shape can refer to the shape of a section that is substantially parallel to the lower surface of the base 120 and/or substantially parallel to the skin surface when the protrusions 110 are contacting the skin surface. Other cross-sectional shapes may be used, such as square, rectangular, hexagonal, ovoid, and the like. Further, the cross-sectional shape of the distal or tip portion of the protrusions 110 can have a different cross-sectional shape than other portions of the protrusions 110. For example, the distal end of a protrusion 110 can be provided with a substantially circular cross-sectional shape, whereas a proximal portion of the protrusion 110 can be square or rectangular in cross section. The distal portion of the protrusions 110 can have cross-sectional shapes, for example, that are round, square, polygonal, ovoid, or irregular. Other shapes can also be used. The ...
http://www.patentsencyclopedia.com/app/20110313411
GRISCELLI SYNDROME, TYPE 2 | Semantic ScholarGRISCELLI SYNDROME, TYPE 2 | Semantic Scholar
A rare, autosomal recessive genetic syndrome caused by mutations in the RAB27A gene. It is characterized by hypopigmentation of the skin, hair and eyes, recurrent infections, neutropenia, and immune system abnormalities. Patients are prone to develop hemophagocytic lymphohistiocytosis.
https://www.semanticscholar.org/topic/GRISCELLI-SYNDROME%2C-TYPE-2/462259
Depigmentation Cases Prompt New Label Warning for ADHD PatchDepigmentation Cases Prompt New Label Warning for ADHD Patch
After dozens of reports of skin depigmentation or hypopigmentation associated with use of the methylphenidate transdermal system marketed as Daytrana, FDA officials are requiring that a new warning be added to the medications package label.
https://www.aafp.org/news/health-of-the-public/20150626daytrana.html
Vitiligo following intense pulsed light treatment<...Vitiligo following intense pulsed light treatment<...
TY - JOUR. T1 - Vitiligo following intense pulsed light treatment. AU - Shin, Jung U.. AU - Roh, Mi Ryung. AU - Lee, Ju Hee. PY - 2010/7. Y1 - 2010/7. N2 - Vitiligo is an acquired depigmenting disorder characterized by the progressive loss of melanocytes from the epidermis and epidermal appendages, which results in milky-white macular lesions. Various factors are suspected to affect the induction and progression of vitiligo such as emotional shock, sunburn, pregnancy, physical illness and trauma. The intense pulsed light (IPL) device which mostly affects redness and dyspigmentation has a broad spectrum of emissions of white light with wavelengths between approximately 515 and 1200 nm. Adverse effects such as purpura and pigmentary changes are known to be rare. We present a 41-year-old woman who developed multiple round, hypopigmented macules on both the cheek and mandibular area following the treatment with IPL for lentigines and dyspigmentation. Based on biopsy and Woods lamp examination, ...
https://yonsei.pure.elsevier.com/en/publications/vitiligo-following-intense-pulsed-light-treatment
How does ReLume Repigmentation Phototherapy work?How does ReLume Repigmentation Phototherapy work?
The ReLume systems unique light-based therapy improves the appearance of leukoderma by pigmenting affected skin. The precise delivery of therapeutic light enhances melanin production to darken the lesion so it more closely resembles the surrounding healthy skin. 6 12 weekly treatments are routinely needed to restore 75% or more of the lost pigment. The number of treatments required may vary according to the origin of the hypopigmentation and skin type. Prolonged improvement can be achieved through maintenance treatments.. ...
http://www.experts123.com/q/how-does-relume-repigmentation-phototherapy-work.html
International Mammalian Genome Society 1999 AbstractsInternational Mammalian Genome Society 1999 Abstracts
D14 Genomic Structure of the Mouse Ap3b1 Gene. Brian W. Rigatti1, Lijun Feng2, Edward, K. Novak3, Richard T. Swank2 and Michael B. Gorin3. Department of Ophthalmology, University of Pittsburgh, Pittsburgh, PA 15213; 2Department of Molecular and Cell Biology, Roswell Park Cancer Institute, Elm and Carlton Streets, Buffalo, NY 14263; 3Departments of Human Genetics and Ophthalmology, University of Pittsburgh, Pittsburgh, PA 15213 The pearl (pe) mouse is a hypopigmentation mutant and a model for human Hermansky-Pudlak Syndrome (HPS). It has been determined that the pearl phenotype arises from mutation in the Ap3b1 gene, which encodes the b3A subunit of the AP-3 adaptor complex, which in turn regulates vesicular trafficking. It has been demonstrated that depletion of the b3A subunit in the pearl mouse results in the depletion of the whole AP-3 adaptor complex which in turn leads to abnormalities of biogenesis/function of subcellular organelles like lysosomes, melanosomes and platelet dense granules. ...
http://www.imgs.org/Archive/abstracts/99abstracts/feng.shtml
Pigmentation ProblemsPigmentation Problems
Pigmentation problems and some skin conditions occur because the body produces either too much melanin (hyperpigmentation) or too little (hypopigmentation), which is the natural pigment responsible for the colour of our skin, hair and eyes.
http://www.dermatologysydney.com.au/category/pigmentation-problems/
Everything about skin tighteningEverything about skin tightening
We have not professional any substantial or lengthy-lasting adverse consequences and also have we found a lower incidence of article-inflammatory hyperpigmentation and no noted instances of hypopigmentation. The medical endpoint we try to find with Fraxel Laser Treatment method is mobile turnover and peeling. Immediately after cure, the affected persons skin tends to be pinkish-pink for a few days with slight swelling instantly soon after remedy ...
http://dallasidysm.tribunablog.com/everything-about-skin-tightening-3506040
mental deficits | Hereditary Ocular Diseasesmental deficits | Hereditary Ocular Diseases
This disorder has a wide variety of clinical features and many occur in only a few patients. The skin has focal areas of hypoplasia with hypopigmentation, often appearing in a streak or linear pattern. These areas may be present at birth and contain bullae or urticarial lesions with signs of inflammation. Telangiectases and herniated fat may appear in these areas. Oral, esophageal, and laryngeal fibrovascular papillomas occur but they may also be seen in the perineal, vulvar, and perianal areas. These may be large, friable, and recurrent. The teeth erupt late and are usually hypoplastic. The nails are often dysplastic and the hands and feet may be split with syndactyly of the third and fourth fingers giving a lobster claw appearance. Polydactyly may be present. Most have thin protruding ears. A variety of skeletal anomalies have been reported including absence of metatarsals and metacarpals. A considerable number of patients have mild to moderate mental deficits. Severely affected females ...
https://disorders.eyes.arizona.edu/category/clinical-features/mental-deficits
bcl-2 deficiency in mice leads to pleiotropic abnormalities: accelerated lymphoid cell death in thymus and spleen, polycystic...bcl-2 deficiency in mice leads to pleiotropic abnormalities: accelerated lymphoid cell death in thymus and spleen, polycystic...
Kamada S., Shimono A., Shinto Y., Tsujimura T., Takahashi T., Noda T., Kitamura Y., Kondoh H., Tsujimoto Y.. Transgenic mice homozygously lacking in the bcl-2 gene were generated using homologous recombination in embryonal stem cells. The complete absence of Bcl-2 alpha and -beta proteins did not interfere with normal embryonic development. Abnormalities became evident after birth, although the severity varied among homozygous null mice, bcl-2-/-mice displayed pleiotropic abnormalities similar to those in the previously described bcl-2-/-mice, including growth retardation, smaller ears, short lives, polycystic kidney, atrophic thymus and spleen with accelerated apoptotic cell death of lymphocytes, and hair hypopigmentation in the second hair follicle cycle. Our bcl-2-/-mice also revealed novel defects in the small intestine, characterized by retarded development, accelerated exfoliation of epithelial cells, and very few mitotic progenitor cells.. Cancer Res. 55:354-359(1995) [PubMed] [Europe ...
http://www.uniprot.org/citations/7812968
Getting My Mehndi Design Video To WorkGetting My Mehndi Design Video To Work
Normally wash your fingers with cleaning soap and h2o prior to deciding to apply henna. You can even use Liquor centered lotion if your skin is oily, and to get the dark colour with your handsyou can implement mehendi oil in addition. The U.S. Food and Drug Administration (FDA) dont advise henna to be used right over the pores and skin. There are reviews of Speak to dermatitis, an allergy a result of the pigment existing in the dye. Discolouration from the skin or Hypopigmentation has also been documented. So in America it is actually used as a hair dye ...
http://andersonwnfw269360.tribunablog.com/getting-my-mehndi-design-video-to-work-3510900
Vitalia MedSpa | Palomar Icon™ Aesthetic System - Vitalia MedSpaVitalia MedSpa | Palomar Icon™ Aesthetic System - Vitalia MedSpa
How does laser skin resurfacing work?. Its all about using beams of light. Your licensed and certified provider uses the laser to send short, concentrated pulsating beams of light at irregular skin. This removes unwanted, damaged skin in a very precise manner one layer at a time.. The laser beam used in laser resurfacing removes the outer layer of skin, called the epidermis. It simultaneously heats the underlying skin, called the dermis. This action works to stimulate growth of new collagen fibers. As the treated area heals, the new skin that forms is smoother and firmer.. Laser skin resurfacings targeted approach means there are fewer problems with hypopigmentation, or a lightening of skin for procedures such as laser acne scar reduction.. Ablative Lasers vs. Non-Ablative Lasers. In general, laser resurfacing treatments fall under one of two categories: ablative lasers and non-ablative lasers. Both ablative and non-ablative resurfacing can give you dramatic improvements, but they do differ ...
https://vitaliamedspava.com/palomar-icon-aesthetic-system/
Abnormally dark or light skin | UF Health, University of Florida HealthAbnormally dark or light skin | UF Health, University of Florida Health
DefinitionAbnormally dark or light skin is skin that has turned darker or lighter than normal.Alternative NamesHyperpigmentation; Hypopigmentation; Skin - abnormally light or dark
https://m.ufhealth.org/abnormally-dark-or-light-skin
Treating Lip Lines | Resurfacing Perfected by Sciton | ResurfacingPerfected.comTreating Lip Lines | Resurfacing Perfected by Sciton | ResurfacingPerfected.com
Erase perioral lines completely and safely with the Sciton Tunable Resurfacing Laser. With the Tunable Resurfacing Laser, you can achieve exceptional results without side effects like hypopigmentation, sustained erythema, and prolonged recovery times. I think everyone would agree that the most challenging lines in the face are perioral wrinkles. In my opinion, Scitons full field tunable laser is by far the best choice because it goes deeper than other resurfacing solutions and eliminates perioral wrinkles without prolonged downtime. The Sciton tunable resurfacing laser allows you to safely manage patient results ...
http://resurfacingperfected.com/versatility/lips
Tanning Beds/Tanning Booths - Bethesda, MD Dermatologist
According to the American Academy of Dermatology and the U.S. Department of Health and Human Services, ultraviolet (UV) radiation from tanning beds, tanning booths and sun lamps are known carcinogens (cancer-causing substances). Exposure to UV radiation during indoor tanning has been proven to increase the risk of all skin cancers, including melanomas, squamous cell carcinomas and basal cell carcinomas. In fact, the risk of melanoma increases by 75 percent when indoor tanning devices are used before the age of 30. The UV radiation during indoor tanning also leads to skin aging, hyper - and hypopigmentation, immune suppression and eye damage, such as cataracts.. Therefore, the use of tanning beds, tanning booths and sun lamps is not recommended by dermatologists.. ...
https://www.chevychasedermatologycenter.com/library/4244/TanningBedsTanningBooths.html
Tanning Beds/Tanning Booths - Santa Monica, CA Dermatologist
According to the American Academy of Dermatology and the U.S. Department of Health and Human Services, ultraviolet (UV) radiation from tanning beds, tanning booths and sun lamps are known carcinogens (cancer-causing substances). Exposure to UV radiation during indoor tanning has been proven to increase the risk of all skin cancers, including melanomas, squamous cell carcinomas and basal cell carcinomas. In fact, the risk of melanoma increases by 75 percent when indoor tanning devices are used before the age of 30. The UV radiation during indoor tanning also leads to skin aging, hyper - and hypopigmentation, immune suppression and eye damage, such as cataracts.. Therefore, the use of tanning beds, tanning booths and sun lamps is not recommended by dermatologists.. ...
https://www.santamonicadermatologygroup.com/library/4244/TanningBedsTanningBooths.html
Tanning Beds/Tanning Booths - Daytona Beach, FL Dermatologist
According to the American Academy of Dermatology and the U.S. Department of Health and Human Services, ultraviolet (UV) radiation from tanning beds, tanning booths and sun lamps are known carcinogens (cancer-causing substances). Exposure to UV radiation during indoor tanning has been proven to increase the risk of all skin cancers, including melanomas, squamous cell carcinomas and basal cell carcinomas. In fact, the risk of melanoma increases by 75 percent when indoor tanning devices are used before the age of 30. The UV radiation during indoor tanning also leads to skin aging, hyper - and hypopigmentation, immune suppression and eye damage, such as cataracts.. Therefore, the use of tanning beds, tanning booths and sun lamps is not recommended by dermatologists.. ...
http://www.hellerdermcenter.net/library/4244/TanningBedsTanningBooths.html
Tanning Beds/Tanning Booths - Dermatologist in Annapolis, MDTanning Beds/Tanning Booths - Dermatologist in Annapolis, MD
According to the American Academy of Dermatology and the U.S. Department of Health and Human Services, ultraviolet (UV) radiation from tanning beds, tanning booths and sun lamps are known carcinogens (cancer-causing substances). Exposure to UV radiation during indoor tanning has been proven to increase the risk of all skin cancers, including melanomas, squamous cell carcinomas and basal cell carcinomas. In fact, the risk of melanoma increases by 75 percent when indoor tanning devices are used before the age of 30. The UV radiation during indoor tanning also leads to skin aging, hyper - and hypopigmentation, immune suppression and eye damage, such as cataracts.. Therefore, the use of tanning beds, tanning booths and sun lamps is not recommended by dermatologists.. ...
https://www.newmanderm.com/library/4244/TanningBedsTanningBooths.html
Swinfen Charitable Trust | Just another WordPress site | Page 8Swinfen Charitable Trust | Just another WordPress site | Page 8
A 6 month old infant was seen by doctors on the 16th July at one of our mobile medical clinics in rural Zambia. The rash developed 1 week before attending clinic. Some lesions were crusting and the rash was present on the face, arms, back and buttocks. The rash did not appear to be bothering the infant and there was no fever or systemic symptoms. The infant was otherwise fit and well, HIV negative and had been seen previously by our doctors at which time there was no evident rash. This rash had been treated by the community health worker with IM benzylpenicillin with some effect.. We sent the case to a Professor specialising in Paediatric Dermatology from the USA.. This very prominent facial rash with an overall annular scaly appearance and impressive hypopigmentation is characteristic of NEONATAL LUPUS ERYTHEMATOSIS, a rare disorder in which infants have maternal antibodies (SS-A, SS-B sometimes called Ro and La antibodies, rarely RNP antibodies) which are photosensitizing so the rash appears ...
https://www.swinfencharitabletrust.org/news/page/8/
HypopigmentationHypopigmentation
Often, hypopigmentation can be brought on by laser treatments; however, the hypopigmentation can be treated with other lasers ... Hypopigmentation can be upsetting to some, especially those with darker skin whose hypopigmentation marks are seen more visibly ... these are the commonly affected areas in those with hypopigmentation. Hypopigmentation is common and approximately one in ... Most causes of hypopigmentation are not serious and can be easily treated. It is seen in: Albinism Idiopathic guttate ...
https://en.wikipedia.org/wiki/Hypopigmentation
Postinflammatory hypopigmentationPostinflammatory hypopigmentation
... is a cutaneous condition characterized by decreased pigment in the skin following ...
https://en.wikipedia.org/wiki/Postinflammatory_hypopigmentation
Yemenite deaf-blind hypopigmentation syndromeYemenite deaf-blind hypopigmentation syndrome
... is a condition caused by a mutation on the SRY-related HMG-box gene 10 (not SOX10 ... September 1999). "A molecular analysis of the yemenite deaf-blind hypopigmentation syndrome: SOX10 dysfunction causes different ... September 1990). "The Yemenite deaf-blind hypopigmentation syndrome. A new oculo-dermato-auditory syndrome". Ophthalmic ... "Yemenite deaf-blind hypopigmentation syndrome". Online Mendelian Inheritance in Man. Johns Hopkins University. Retrieved 25 ...
https://en.wikipedia.org/wiki/Yemenite_deaf-blind_hypopigmentation_syndrome
National Organization for Albinism and HypopigmentationNational Organization for Albinism and Hypopigmentation
The National Organization for Albinism and Hypopigmentation (NOAH) is a non-profit organization that assists people who have ... healthfinder.gov - National Organization for Albinism and Hypopigmentation - NOAH News: 'Da Vinci Code' premier sparks protests ...
https://en.wikipedia.org/wiki/National_Organization_for_Albinism_and_Hypopigmentation
Albinism in humansAlbinism in humans
"About Albinism and Hypopigmentation , National Organization for Albinism and Hypopigmentation". 15 December 2020. "Ukerewe ... "Information Bulletin - Ocular Albinism". National Organization for Albinism and Hypopigmentation. Archived from the original on ... According to the National Organization for Albinism and Hypopigmentation, "With ocular albinism, the color of the iris of the ...
https://en.wikipedia.org/wiki/Albinism_in_humans
Nevus depigmentosusNevus depigmentosus
Pinto, FJ; Bolognia, JL (1991). "Disorders of hypopigmentation in children". Pediatric Clinics of North America. U.S. National ...
https://en.wikipedia.org/wiki/Nevus_depigmentosus
PiebaldPiebald
Introduction to Hypopigmentation; Chapter 147, Albinism; Chapter 148, Piebaldism; Chapter 149, Vitiligo. Spontaneous Animal ...
https://en.wikipedia.org/wiki/Piebald
Pityriasis albaPityriasis alba
The hypopigmentation is due to both reduced activity of melanocytes with fewer and smaller melanosomes. The cause of pityriasis ... ISBN 978-1-4051-5733-9. Pinto FJ, Bolognia JL (August 1991). "Disorders of hypopigmentation in children". Pediatric Clinics of ...
https://en.wikipedia.org/wiki/Pityriasis_alba
Genetic studies on ArabsGenetic studies on Arabs
Yemenite deaf-blind hypopigmentation syndrome (1990) •• Nablus mask-like facial syndrome (2000) •• Jerash type of the distal ...
https://en.wikipedia.org/wiki/Genetic_studies_on_Arabs
AlbinismAlbinism
"Information Bulletin - What is Albinism?". National Organization for Albinism and Hypopigmentation. January 27, 2018. Retrieved ...
https://en.wikipedia.org/wiki/Albinism
Oral pigmentationOral pigmentation
Schaffer JV, Bolognia JL (2003). "The treatment of hypopigmentation in children". Clinics in Dermatology. 21 (4): 296-310. doi: ...
https://en.wikipedia.org/wiki/Oral_pigmentation
Persecution of people with albinismPersecution of people with albinism
Organisations such as National Organization for Albinism and Hypopigmentation (NOAH), Tanzania Albino Centre (TAC), based in ... Accessed 15 April 2014 "National Organization for Albinism and Hypopigmentation". National Organization for Albinism and ... Hypopigmentation. Retrieved 3 March 2020. Tanzania Albino Center (TAC) http://www.tanzaniaalbino.org/ Archived 7 May 2014 at ...
https://en.wikipedia.org/wiki/Persecution_of_people_with_albinism
Cross syndromeCross syndrome
"Orphanet: Oculocerebral hypopigmentation syndrome, Cross type". www.orpha.net. Retrieved 19 April 2019. James, William; Berger ... ISBN 0-7216-2921-0. Cross HE, McKusick VA, Breen W (March 1967). "A new oculocerebral syndrome with hypopigmentation". J. ... ", "hypopigmentation and microphthalmia", and "oculocerebral-hypopigmentation syndrome") is an extremely rare disorder ...
https://en.wikipedia.org/wiki/Cross_syndrome
White horseWhite horse
"What is Albinism?". The National Organization for Albinism and Hypopigmentation. Archived from the original on 14 May 2012. ...
https://en.wikipedia.org/wiki/White_horse
International Albinism Awareness DayInternational Albinism Awareness Day
n.a. (n.d.). "History of International Albinism Awareness Day". National Organization of Albinism and Hypopigmentation (NOAH). ... n.a. (n.d.). "History of International Albinism Awareness Day" (PDF). National Organization of Albinism and Hypopigmentation ( ... on International Albinism Awareness Day OHCHR on Albinism National Organization for Albinism and Hypopigmentation (NOAH) in the ...
https://en.wikipedia.org/wiki/International_Albinism_Awareness_Day
Siberian HuskySiberian Husky
This condition is called hypopigmentation in animals. "Snow nose" is acceptable in the show ring. Siberian Husky tails are ...
https://en.wikipedia.org/wiki/Siberian_Husky
Dominant whiteDominant white
The National Organization for Albinism and Hypopigmentation. Archived from the original on 2012-05-14. Retrieved 2009-07-07. ...
https://en.wikipedia.org/wiki/Dominant_white
Tiger eyeTiger eye
Vogel P, Read RW, Vance RB, Platt KA, Troughton K, Rice DS (March 2008). "Ocular albinism and hypopigmentation defects in ...
https://en.wikipedia.org/wiki/Tiger_eye
Tattoo removalTattoo removal
Allowing more time between treatments reduces chances of hypopigmentation. Since it is more likely to see hypopigmentation ... Hypopigmentation is more commonly observed in darker skin tones. It is more likely to occur with higher fluence and more ... Usually treatment stops until hypopigmentation resolves in a matter of months. Transient textural changes are occasionally ... Goldberg, David J. (4 December 2007). "Chapter 3: Pigmented Lesions, Tattoos, and Disorders of Hypopigmentation". Laser ...
https://en.wikipedia.org/wiki/Tattoo_removal
Bioptics (device)Bioptics (device)
East Hampstead, NH: National Organization for Albinism and Hypopigmentation. 2002. Archived from the original on April 3, 2009 ... East Hampstead, NH: National Organization for Albinism and Hypopigmentation. 2002. Archived from the original on April 3, 2009 ...
https://en.wikipedia.org/wiki/Bioptics_(device)
Microphthalmia-associated transcription factorMicrophthalmia-associated transcription factor
Smith SD, Kelley PM, Kenyon JB, Hoover D (June 2000). "Tietz syndrome (hypopigmentation/deafness) caused by mutation of MITF". ...
https://en.wikipedia.org/wiki/Microphthalmia-associated_transcription_factor
HyperpigmentationHyperpigmentation
Hypopigmentation List of skin conditions Drug-induced pigmentation "Hyperpigmentation". Dermatalogic Disease Database. American ...
https://en.wikipedia.org/wiki/Hyperpigmentation
Trichophyton concentricumTrichophyton concentricum
Tinea imbricata has been known to cause hypopigmentation and hyperpigmentation. Susceptibility to this infection has been ...
https://en.wikipedia.org/wiki/Trichophyton_concentricum
Noonan syndrome with multiple lentiginesNoonan syndrome with multiple lentigines
Also, some areas of vitiligo-like hypopigmentation may be observed. Electrocardiographic conduction abnormalities: Generally ... Additional dermatologic abnormalities (axillary freckling, localized hypopigmentation, interdigital webbing, hyperelastic skin ...
https://en.wikipedia.org/wiki/Noonan_syndrome_with_multiple_lentigines
DyschromiaDyschromia
"Hyperchromia" can refer to hyperpigmentation, and "hypochromia" can refer to hypopigmentation. "Dyschromatoses" involve both ...
https://en.wikipedia.org/wiki/Dyschromia
Tumid lupus erythematosusTumid lupus erythematosus
LET resolves with normal skin, no residual scarring, no hyperpigmentation or hypopigmentation. Cigarette smokers who have LET ...
https://en.wikipedia.org/wiki/Tumid_lupus_erythematosus
Tietz syndromeTietz syndrome
... hypopigmentation/deafness) caused by mutation of MITF" (Free full text). J. Med. Genet. 37 (6): 446-448. doi:10.1136/jmg.37.6. ...
https://en.wikipedia.org/wiki/Tietz_syndrome
P14 deficiencyP14 deficiency
Beyond immunodeficiency, patients with p14 deficiency also show hypopigmentation and short stature. Hypopigmentation is caused ... leading to hypopigmentation and a decrease in T-cell cytotoxicity. It is also consistent with the inefficient digestion ...
https://en.wikipedia.org/wiki/P14_deficiency
Dog coatDog coat
This leads to a dry coat, hypo-pigmentation, and discoloration of the coat. Selenium is another one of the many trace minerals ...
https://en.wikipedia.org/wiki/Dog_coat
Incontinentia pigmentiIncontinentia pigmenti
... followed by linear hypopigmentation. Alopecia, dental anomalies, and dystrophic nails are observed. Some patients have retinal ... swirls or streaks of hypopigmentation and depigmentation. It is not inherited and does not involve skin stages 1 or 2. Some 33- ...
https://en.wikipedia.org/wiki/Incontinentia_pigmenti
Teen Spotlight - National Organization for Albinism and HypopigmentationTeen Spotlight - National Organization for Albinism and Hypopigmentation
National Organization for Albinism and Hypopigmentation. Where people with albinism and their families can find acceptance, ...
https://albinism.org/teenspotlight/
Lighthouse Guild Tele-Support - National Organization for Albinism and HypopigmentationLighthouse Guild Tele-Support - National Organization for Albinism and Hypopigmentation
National Organization for Albinism and Hypopigmentation. Where people with albinism and their families can find acceptance, ...
https://www.albinism.org/lg-telesupport/
Intralesional corticosteroid-induced hypopigmentation and atrophyIntralesional corticosteroid-induced hypopigmentation and atrophy
Atrophy and hypopigmentation most commonlyremain localized to sites of injection. However, outward radiation in a linear, ... Intralesional corticosteroids are associated with various, uncommon, local adverse events [1]. Atrophy and hypopigmentation ... "perilesional/perilymphatic hypopigmentation or atrophy [2]." We report a case of this rare adverse event. ...
https://dermatology.cdlib.org/uc/item/0bj81707
Hypopigmentation Skin Overview New York, NY | Laser CenterHypopigmentation Skin Overview New York, NY | Laser Center
Hypopigmentation is the lightening of the skin that may occur after injury to the skin from a procedure or trauma. Various ... Q: What Causes Hypopigmentation?. A: Hypopigmentation occurs when the skin lacks or stops producing melanin. Melanin is a ... Hypopigmentation may occur after injury to the skin from a previous procedure or trauma. It can also a side effect of vitiligo ...
https://www.laserskinsurgery.com/conditions/hypopigmentation/
skin hypopigmentation | Hereditary Ocular Diseasesskin hypopigmentation | Hereditary Ocular Diseases
Hypopigmentation in the scalp hair, eyebrows and eyelashes was more severe than usually seen in patients with single mutations ... Hypopigmentation in the scalp hair, eyebrows and eyelashes was more severe than usually seen in patients with single mutations ... The latter may be partial in individual irides, or the entire iris in one eye with the fundus hypopigmentation often matching ... Dysmorphic features such as micrognathia, microcephaly, low-set ears, some degree of generalized hypopigmentation (hair and ...
https://disorders.eyes.arizona.edu/category/clinical-features/skin-hypopigmentation
IMSEAR at SEARO: Imatinib mesylate induced skin hypopigmentation.
Hypopigmentation --chemically induced. en_US. dc.subject.mesh. Leukemia, Myelogenous, Chronic, BCR-ABL Positive --drug therapy ... Talwar V, Doval DC, Bhatia K. Imatinib mesylate induced skin hypopigmentation. Journal of the Association of Physicians of ...
https://imsear.searo.who.int/handle/123456789/87689?mode=full
Hypopigmentation Treatment - Causes, Treatment, Results And Cost
Hypopigmentation Treatment - Get treatment of an injury or a trauma that the skin has went through. Call our expert ... What Exactly is Hypopigmentation?. Hypopigmentation is a condition where you have patches of skin that are lighter than other ... Hypopigmentation Treatment Answers to Your Hypopigmentation Problems. Have you ever noticed lighter-colored patches of skin ... Successful Hypopigmentation Treatment at Winston Salem Dermatology. Are you fed up with your hypopigmentation? Dont be! Let ...
https://www.winstonsalemdermatology.com/skin-tightening-and-enhancement/hypopigmentation-treatment/
Oculocerebral hypopigmentation syndrome type Preus - Rare Rheumatology NewsOculocerebral hypopigmentation syndrome type Preus - Rare Rheumatology News
PubMed is a searchable database of medical literature and lists journal articles that discuss Oculocerebral hypopigmentation ...
https://www.rarerheumatologynews.com/rarediseases/oculocerebral-hypopigmentation-syndrome-type-preus/
Abnormally dark or light skin: MedlinePlus Medical EncyclopediaAbnormally dark or light skin: MedlinePlus Medical Encyclopedia
Vitiligo and other disorders of hypopigmentation. In: Bolognia JL, Schaffer JV, Cerroni L, eds. Dermatology. 4th ed. ...
https://medlineplus.gov/ency/article/003242.htm
A genome-wide association study reveals a locus for bilateral iridal hypopigmentation in Holstein Friesian cattle | BMC Genomic...A genome-wide association study reveals a locus for bilateral iridal hypopigmentation in Holstein Friesian cattle | BMC Genomic...
... significantly increased the chance of iridal hypopigmentation. The clinical appearance of the iridal hypopigmentation differed ... Iridal hypopigmentation is caused by a reduced content of melanin pigment in the anterior border layer and iridal stroma. A ... Analysis of genotypic and allelic dependences between cases of iridal hypopigmentation and an additional set of 316 randomly ... a reduction of melanin pigment in the iridal anterior border layer and stroma in cases as cause of iris hypopigmentation. To ...
https://bmcgenomdata.biomedcentral.com/articles/10.1186/s12863-017-0496-4
Cartilage-Hair Hypoplasia Clinical Presentation: History, Physical, CausesCartilage-Hair Hypoplasia Clinical Presentation: History, Physical, Causes
Cartilage-hair hypoplasia (CHH), which is Online Mendelian Inheritance in Man (OMIM) disease number 250250, is an autosomal recessive inherited disorder that results in short-limb dwarfism associated with T-cell and B-cell immunodeficiency. Cartilage-hair hypoplasia and other short-limb dwarfism phenotypes are associated with metaphyseal or s...
https://www.medscape.com/answers/885807-191621/what-causes-cartilage-hair-hypoplasia-chh
Feeding difficulties and Hypopigmentation of the skin, related diseases and genetic alterations | MENDELIAN.COFeeding difficulties and Hypopigmentation of the skin, related diseases and genetic alterations | MENDELIAN.CO
FEEDING DIFFICULTIES and HYPOPIGMENTATION OF THE SKIN related symptoms, diseases, and genetic alterations. Get the complete ... Feeding difficulties, and Hypopigmentation of the skin. .site-footer{margin-bottom:80px}. If you liked this article maybe you ... Feeding difficulties and Hypopigmentation of the skin, related diseases and genetic alterations ...
https://www.mendelian.co/symptoms/feeding-difficulties-and-hypopigmentation-of-the-skin
Melasma: Causes, symptoms, pictures & treatmentMelasma: Causes, symptoms, pictures & treatment
What is hypopigmentation?. Some people lose skin pigmentation for a number of reasons. Here, learn about some types of ...
https://www.medicalnewstoday.com/articles/323715?utm_source=ReadNext
Ala Quin Cream: Package Insert / Prescribing Information - Drugs.comAla Quin Cream: Package Insert / Prescribing Information - Drugs.com
... hypopigmentation; perioral dermatitis; allergic contact dermatitis; maceration of the skin, secondary infection; skin atrophy; ...
https://www.drugs.com/pro/ala-quin-cream.html
Syndromic Sensorineural Hearing Loss Clinical Presentation: History, Physical, CausesSyndromic Sensorineural Hearing Loss Clinical Presentation: History, Physical, Causes
Hypopigmentation - Albinism. *. Lentigines - Lentigines, electrocardiographic abnormalities, ocular hypertelorism, pulmonary ...
https://emedicine.medscape.com/article/856116-clinical
Consensus on Treatment of Acne Scars in China (2021)# : International Journal of Dermatology and VenereologyConsensus on Treatment of Acne Scars in China (2021)# : International Journal of Dermatology and Venereology
... and hypopigmentation. Directions for usage: 1.0 mL of compound betamethasone (7 g/L) or triamcinolone acetonide solution (10 g/ ...
https://journals.lww.com/ijdv/Fulltext/2022/09000/Consensus_on_Treatment_of_Acne_Scars_in_China.1.aspx
DailyMed - TRIAMCINOLONE ACETONIDE creamDailyMed - TRIAMCINOLONE ACETONIDE cream
Hypopigmentation. Perioral dermatitis. Allergic contact dermatitis. Maceration of the skin. Secondary infection. Skin Atrophy. ...
https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=557fa0fc-178f-4574-8a88-3c6d8e7cbc9b&audience=professional
Advanced Search Results - Public Health Image Library(PHIL)Advanced Search Results - Public Health Image Library(PHIL)
Categories: Hypopigmentation Image Types: Photo, Illustrations, Video, Color, Black&White, PublicDomain, CopyrightRestricted 4 ...
https://phil.cdc.gov/AdvancedSearchResults.aspx?Search=Hypopigmentation&parentid=20275&catid=31986
Diagnosis and Treatment of Acne | AAFPDiagnosis and Treatment of Acne | AAFP
Hypopigmentation, burning, stinging, tingling, pruritus. Cream (Azelex, 20%; approved for acne vulgaris). NA ($210). ...
https://www.aafp.org/pubs/afp/issues/2012/1015/p734.html
Kala-azar and Post-Kala-azar Dermal Leishmaniasis, Assam, India - Volume 20, Number 3-March 2014 - Emerging Infectious Diseases...
Macular hypopigmentation developed on her face and body 3.5 years after treatment. ...
https://wwwnc.cdc.gov/eid/article/20/3/13-0260_article
Albinism Resources Guide - Disability ResourcesAlbinism Resources Guide - Disability Resources
National Organization for Albinism & Hypopigmentation (NOAH). NOAHs website provides information about albinism and NOAH ...
https://www.disabilityresources.org/albinism.html
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Buy Locoid Cream Online - Universal Drugstore™Buy Locoid Cream Online - Universal Drugstore™
skin hypopigmentation. *central serous chorioretinopathy. *hypothalamic-pituitary insufficiency. *adrenal cortical ...
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Novel skin phenotypes revealed by a genome-wide mouse reverse genetic screen | Nature CommunicationsNovel skin phenotypes revealed by a genome-wide mouse reverse genetic screen | Nature Communications
... which is characterized by hypopigmentation of the skin and hair. Several spontaneous mutations in the dilute locus were ...
https://www.nature.com/articles/ncomms4540?error=cookies_not_supported
Retin A, Renova (tretinoin topical) dosing, indications, interactions, adverse effects, and moreRetin A, Renova (tretinoin topical) dosing, indications, interactions, adverse effects, and more
Medscape - Acne dosing for Retin A, Renova (tretinoin topical), frequency-based adverse effects, comprehensive interactions, contraindications, pregnancy & lactation schedules, and cost information.
http://reference.medscape.com/drug/343555
Skin Care Review: Fraxel Repair CO2 Laser
Scars, hypopigmentation, texture issues. I was in bandages for 5 weeks. Details. Fraxel Repair was not painful, but the healing ... Fraxel has left me with scars, hypo pigmentation, hyper pigmentation and very rough skin. BE CAREFUL. If you would like to see ...
http://www.smartskincare.com/reviews/product/antiaging_invproc_fraxel_repair-co2-laser_20080826.html
Acne Scars Treatment: How to Get Rid of Acne ScarsAcne Scars Treatment: How to Get Rid of Acne Scars
Can result in hypopigmentation. The procedure usually takes place in a dermatologists office after a scar has healed. It takes ...
https://www.drugwatch.com/health/acne/how-to-get-rid-of-scars/
Dermoscopy. Pattern analysis | DermNetDermoscopy. Pattern analysis | DermNet
Central hypopigmentation. *Symmetrical blotches. *Comma-like regular vasculature. *On face: regular pseudonetwork ...
https://dermnetnz.org/cme/dermoscopy-course/pattern-analysis
HyperpigmentationNational Organization forOculocutaneous hypopigmentationPigmentationAtrophySkinMelaninScarsDisordersHyperCasesTreatmentJournalCentralFaceChance
Hyperpigmentation3
  • Hyperpigmentation (dark spots) or hypopigmentation (light spots). (richardswiftmd.com)
  • One case of hyperpigmentation and one case of hypopigmentation were observed in the NSL treatment group. (bvsalud.org)
  • Side effects may include redness, swelling, hyperpigmentation, hypopigmentation, blistering and purpura (a laser bruise) in the treated area-but these usually do not last more than a few days. (tecumsehlasercentre.com)
National Organization for2
  • This article describes an event hosted by the author to bring those in the albinism community together on behalf of NOAH, the National Organization for Albinism and Hypopigmentation. (tsbvi.edu)
  • For the last 14 years, the National Organization for Albinism and Hypopigmentation (NOAH) has rallied its members to participate in an annual fundraising event. (tsbvi.edu)
Oculocutaneous hypopigmentation1
  • Affected cattle showed an oculocutaneous hypopigmentation (OH) with a pale blue iris and a tan periphery coupled with a change in coat color from black to chocolate. (biomedcentral.com)
Pigmentation1
  • The clinical appearance of the iridal hypopigmentation differed from previously reported cases of pigmentation abnormalities in syndromes like Chediak-Higashi or Tietz and seems to be mainly of cosmetic character. (biomedcentral.com)
Atrophy2
  • Atrophy and hypopigmentation most commonlyremain localized to sites of injection. (cdlib.org)
  • However, outward radiation in a linear, streaky pattern has been reported and is termed "perilesional/perilymphatic hypopigmentation or atrophy [2]. (cdlib.org)
Skin10
  • Hypopigmentation is the lightening of the skin. (laserskinsurgery.com)
  • Hypopigmentation may occur after injury to the skin from a previous procedure or trauma. (laserskinsurgery.com)
  • A: Hypopigmentation occurs when the skin lacks or stops producing melanin. (laserskinsurgery.com)
  • Dysmorphic features such as micrognathia, microcephaly, low-set ears, some degree of generalized hypopigmentation (hair and skin), and a broad nose with a long philtrum may be present. (arizona.edu)
  • IMSEAR at SEARO: Imatinib mesylate induced skin hypopigmentation. (who.int)
  • Talwar V, Doval DC, Bhatia K. Imatinib mesylate induced skin hypopigmentation. (who.int)
  • Hypopigmentation is a condition where you have patches of skin that are lighter than other parts of your body's overall skin tone. (winstonsalemdermatology.com)
  • Hypopigmentation is the loss of that pigment in the skin. (winstonsalemdermatology.com)
  • Griscelli syndrome (GS) is a rare cutaneous disease characterized by a silvery-gray sheen of the hair and hypopigmentation of the skin, which can be associated to primary neurological impairment (type 1), immunologic impairment (type 2) or be isolated (type 3). (nih.gov)
  • If your client says their skin tends to heal lighter or darker, this could be a sign of hyper- or hypopigmentation and cosmetic tattooing may not be a good fit for this client. (darklab.com)
Melanin2
  • Histological examination revealed a reduction of melanin pigment in the iridal anterior border layer and stroma in cases as cause of iris hypopigmentation. (biomedcentral.com)
  • Iridal hypopigmentation is caused by a reduced content of melanin pigment in the anterior border layer and iridal stroma. (biomedcentral.com)
Scars1
Disorders2
  • Vitiligo and other disorders of hypopigmentation. (medlineplus.gov)
  • CYM upregulated Mitf and possibly activates tyrosinase enzyme, providing evidence for its possible use to promote melanogenesis and as a therapeutic agent against hypopigmentation disorders. (hindawi.com)
Hyper1
  • Cream and acid treatments usually take at least 2-6 weeks to resolve and may cause hyper or hypopigmentation in the area treated. (womensclinicla.com)
Cases1
  • Analysis of genotypic and allelic dependences between cases of iridal hypopigmentation and an additional set of 316 randomly selected Holstein Friesian cattle controls showed that allele A at position 60,990,733 on BTA8 ( P = 4.0e-08, odds ratio = 6.3, 95% confidence interval 3.02-13.17) significantly increased the chance of iridal hypopigmentation. (biomedcentral.com)
Treatment1
  • Macular hypopigmentation developed on her face and body 3.5 years after treatment. (cdc.gov)
Journal1
  • PubMed is a searchable database of medical literature and lists journal articles that discuss Oculocerebral hypopigmentation syndrome type Preus. (rarerheumatologynews.com)
Central1
  • A 90-day study in 150 older adults found that supplementing with goji berries increased blood antioxidant levels, prevented hypopigmentation of the macula (the central area of the retina), and helped soften fatty protein deposits under the retina ( 14 ). (healthline.com)
Face1
  • Linear scleroderma involving the face, forehead, and scalp is termed "scleroderma en coup de sabre. (medscape.com)
Chance1
Abnormally dark or light skin: MedlinePlus Medical Encyclopedia
Abnormally dark or light skin: MedlinePlus Medical Encyclopedia (medlineplus.gov)
Cartilage-Hair Hypoplasia Clinical Presentation: History, Physical, Causes
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Guidance for Using Tafenoquine for Prevention and Antirelapse Therapy for Malaria - United States, 2019 | MMWR
Guidance for Using Tafenoquine for Prevention and Antirelapse Therapy for Malaria - United States, 2019 | MMWR (cdc.gov)
Griscelli syndrome: MedlinePlus Genetics
Griscelli syndrome: MedlinePlus Genetics (medlineplus.gov)
Skin, Hair and Nails: MedlinePlus
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Seborrheic Dermatitis: Practice Essentials, Background, Pathophysiology
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Epilepsy and Seizures: Practice Essentials, Background, Pathophysiology
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Chediak-Higashi Syndrome Clinical Presentation: History, Physical Examination, Complications
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Seborrheic Dermatitis Clinical Presentation: History, Physical Examination
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Epilepsy and Seizures: Practice Essentials, Background, Pathophysiology
Epilepsy and Seizures: Practice Essentials, Background, Pathophysiology (medscape.com)
Vitiligo: Practice Essentials, Background, Pathophysiology
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Pityriasis Rosea Clinical Presentation: History, Physical Examination
Pityriasis Rosea Clinical Presentation: History, Physical Examination (medscape.com)
Vitiligo Differential Diagnoses
Vitiligo Differential Diagnoses (medscape.com)
(emedicine.medscape.com)
Complications of Dermatologic Laser Surgery: Overview, Laser Surgery Principles and Potential Complications, Introduction to...
Complications of Dermatologic Laser Surgery: Overview, Laser Surgery Principles and Potential Complications, Introduction to... (emedicine.medscape.com)
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Diagnostic Errors in Patients With Skin Findings
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Laser Eyelid Tissue Resurfacing Treatment & Management: Medical Therapy, Preoperative Details, Intraoperative Details
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