Hyperplasia
Prostatic Hyperplasia
Endometrial Hyperplasia
Adrenal Hyperplasia, Congenital
Gingival Hyperplasia
Thymus Hyperplasia
Focal Nodular Hyperplasia
Tunica Intima
Focal Epithelial Hyperplasia
Neointima
Angiolymphoid Hyperplasia with Eosinophilia
Steroid 21-Hydroxylase
Precancerous Conditions
Pseudolymphoma
Prostate
Parathyroid Glands
Immunohistochemistry
Endometrial Neoplasms
Transurethral Resection of Prostate
Carotid Artery Injuries
17-alpha-Hydroxyprogesterone
Jugular Veins
Goblet Cells
Giant Lymph Node Hyperplasia
Disease Models, Animal
Mice, Transgenic
Parathyroid Diseases
Adrenocortical Hyperfunction
5-alpha Reductase Inhibitors
Cell Division
Hypertrophy
Polytetrafluoroethylene
Papilloma
Vascular Grafting
Cushing Syndrome
Carotid Arteries
Adrenal Glands
Serenoa
Finasteride
Epidermis
Carcinoma in Situ
Myocytes, Smooth Muscle
Endometrium
Angioplasty, Balloon
Carcinogens
Azasteroids
Proliferating Cell Nuclear Antigen
Carcinogenicity Tests
Iliac Artery
Epithelium
Hyperparathyroidism
Breast
Mice, Knockout
Rats, Sprague-Dawley
Biopsy
Fibrocystic Breast Disease
Metaplasia
Adenomatosis, Pulmonary
Urinary Bladder Neck Obstruction
Catheterization
Cells, Cultured
Enterochromaffin Cells
Carotid Artery, Common
Pregnanetriol
Adrenocorticotropic Hormone
Polyps
Rabbits
Skin
Hyperaldosteronism
Anastomosis, Surgical
Blood Vessel Prosthesis
Ultrasonography, Interventional
Stents
RNA, Messenger
Hydroxyprogesterones
Prostatitis
Immunoenzyme Techniques
Lung
Arteriovenous Shunt, Surgical
Cell Transformation, Neoplastic
Carcinoma, Intraductal, Noninfiltrating
Models, Animal
Biopsy, Needle
Citrobacter rodentium
Angiomatosis
Hyperparathyroidism, Secondary
Lower Urinary Tract Symptoms
Constriction, Pathologic
Liver
The epizootiology and pathogenesis of thyroid hyperplasia in coho salmon (Oncorhynchus kisutch) in Lake Ontario. (1/4620)
The thyroid glands of coho salmon collected at different stages of their anadromous migration exhibited progressive and extensive hyperplasia and hypertrophy. The incidence of overt nodule formation rose from 5% in fish collected in August to 24% in fish collected in October. The histological picture of the goiters was similar to that found in thiourea-treated teleosts and thiouracil-treated mammals. There was a concomitant, significant decrease in serum thyroxine and triiodothyronine values between September and October (thyroxine, 1.0+/-0.3 mug/100 ml and 0.4 mug/100 ml in September and October, respectively; triiodothyronine, 400.3+/-51.6 ng/100 ml and 80.2 ng/100 ml in September and October, respectively) and marked hypertrophy and hyperplasia of thyrotrophs. These data indicate a progressive hypothyroid condition which, although it may be linked to iodide deficiency, may well be enhanced by other environmental factors. The evidence for involvement of other factors is discussed. (+info)Natural history of papillary lesions of the urinary bladder in schistosomiasis. (2/4620)
Variable epithelial hyperplasia was observed in urinary bladder of nine capuchin monkeys (Cebus apella) when examined at cystotomy 94 to 164 weeks after infection with Schistosoma haematobium. These hosts were followed for 24 to 136 weeks postcystotomy to determine the status of bladder lesions in relation to duration of infection and to ascertain whether lesion samples removed at cystotomy reestablished themselves in autologous and heterologous transfers. There was involution of urothelial hyperplasia in eight of nine animals and no evidence for establishment of transplanted bladder lesions. (+info)Ganglioneuromas and renal anomalies are induced by activated RET(MEN2B) in transgenic mice. (3/4620)
Multiple endocrine neoplasia type 2B (MEN2B) is an autosomal dominant syndrome characterized by the development of medullary thyroid carcinoma, pheochromocytomas, musculoskeletal anomalies and mucosal ganglioneuromas. MEN2B is caused by a specific mutation (Met918-->Thr) in the RET receptor tyrosine kinase. Different mutations of RET lead to other conditions including MEN2A, familial medullary thyroid carcinoma and intestinal aganglionosis (Hirschsprung disease). Transgenic mice were created using the dopamine beta-hydroxylase promoter to direct expression of RET(MEN2B) in the developing sympathetic and enteric nervous systems and the adrenal medulla. DbetaH-RET(MEN2B) transgenic mice developed benign neuroglial tumors, histologically identical to human ganglioneuromas, in their sympathetic nervous systems and adrenal glands. The enteric nervous system was not affected. The neoplasms in DbetaH-RET(MEN2B) mice were similar to benign neuroglial tumors induced in transgenic mice by activated Ras expression under control of the same promoter. Levels of phosphorylated MAP kinase were not increased in the RET(MEN2B)-induced neurolglial proliferations, suggesting that alternative pathways may play a role in the pathogenesis of these lesions. Transgenic mice with the highest levels of DbetaH-RET(MEN2B) expression, unexpectedly developed renal malformations analogous to those reported with loss of function mutations in the Ret gene. (+info)Anti-monocyte chemoattractant protein-1/monocyte chemotactic and activating factor antibody inhibits neointimal hyperplasia in injured rat carotid arteries. (4/4620)
Monocyte chemoattractant protein-1 (MCP-1)/monocyte chemotactic and activating factor (MCAF) has been suggested to promote atherogenesis. The effects of in vivo neutralization of MCP-1 in a rat model were examined in an effort to clarify the role of MCP-1 in the development of neointimal hyperplasia. Competitive polymerase chain reaction analysis revealed maximum MCP-1 mRNA expression at 4 hours after carotid arterial injury. Increased immunoreactivities of MCP-1 were also detected at 2 and 8 hours after injury. Either anti-MCP-1 antibody or nonimmunized goat IgG (10 mg/kg) was then administered every 12 hours to rats that had undergone carotid arterial injury. Treatment with 3 consecutive doses of anti-MCP-1 antibody within 24 hours (experiment 1) and every 12 hours for 5 days (experiment 2) significantly inhibited neointimal hyperplasia at day 14, resulting in a 27.8% reduction of the mean intima/media ratio (P<0.05) in experiment 1 and a 43.6% reduction (P<0.01) in experiment 2. This effect was still apparent at day 56 (55.6% inhibition; P<0.05). The number of vascular smooth muscle cells in the neointima at day 4 was significantly reduced by anti-MCP-1 treatment, demonstrating the important role of MCP-1 in early neointimal lesion formation. However, recombinant MCP-1 did not stimulate chemotaxis of vascular smooth muscle cells in an in vitro migration assay. These results suggest that MCP-1 promotes neointimal hyperplasia in early neointimal lesion formation and that neutralization of MCP-1 before, and immediately after, arterial injury may be effective in preventing restenosis after angioplasty. Further studies are needed to clarify the mechanism underlying the promotion of neointimal hyperplasia by MCP-1. (+info)Myometrial zonal differentiation and uterine junctional zone hyperplasia in the non-pregnant uterus. (5/4620)
Human non-gravid myometrium differentiates in response to ovarian sex steroids into a subendometrial layer or junctional zone and an outer myometrial layer. Compared to the outer myometrial layer, the junctional zone myocytes are characterized by higher cellular density and lower cytoplasmic-nuclear ratio. These structural differences allow in-vivo visualization of the myometrial zonal anatomy by T2-weighted magnetic resonance (MR) imaging. The human myometrium is also functionally polarized. Video-vaginosonography studies have shown that propagated myometrial contractions in the non-pregnant uterus originate only from the junctional zone and that the frequency and orientation of these contraction waves are dependent on the phase of the menstrual cycle. The mechanisms underlying zonal myometrial differentiation are not known, but growing evidence suggests that ovarian hormone action may be mediated through cytokines and uterotonins locally released by the basal endometrial layer and endometrio-myometrial T-lymphocytes. Irregular thickening of the junctional zone due to inordinate proliferation of the inner myometrium, junctional zone hyperplasia, is a common MR finding in women suffering from menstrual dysfunction. Preliminary data suggest that junctional zone hyperplasia is further characterized by loss of normal inner myometrial function. Although irregular thickening of the junctional zone has been associated with diffuse uterine adenomyosis, the precise relationship between subendometrial smooth muscle proliferation and myometrial invasion by endometrial glands and stroma remains to be established. (+info)Mechanism of parathyroid tumourigenesis in uraemia. (6/4620)
Clonal analysis has shown that in renal hyperparathyroidism (2-HPT), parathyroid glands initially grow diffusely and polyclonally after which the foci of nodular hyperplasia are transformed to monoclonal neoplasia. There is a great deal of information about genetic abnormalities contributing to the tumourigenesis of parathyroid neoplasia in primary hyperparathyroidism. It is speculated that allelic loss of the MEN1 suppressor gene and overexpression of cyclin D1 induced by rearrangement of the parathyroid hormone gene may be the major genetic abnormality in sporadic parathyroid adenoma but not in 2-HPT. The pathogenesis of 2-HPT, abnormality of the Ca2+-sensing receptor (CaR) gene and the vitamin D receptor gene may possibly contribute to parathyroid tumourigenesis in 2-HPT. However, this is not yet clear and heterogeneous and multiple genetic abnormalities may be responsible for the progression of secondary parathyroid hyperplasia. (+info)Accelerated intimal hyperplasia and increased endogenous inhibitors for NO synthesis in rabbits with alloxan-induced hyperglycaemia. (7/4620)
1. We examined whether endogenous inhibitors of NO synthesis are involved in the augmentation of intimal hyperplasia in rabbits with hyperglycaemia induced by alloxan. 2. Four weeks after the endothelial denudation of carotid artery which had been performed 12 weeks after alloxan, the intimal hyperplasia was greatly augmented with hyperglycaemia. The degree of hyperplasia was assessed using three different parameters of histopathological findings as well as changes in luminal area and intima: media ratio. 3. There were positive and significant correlations between intima:media ratio, plasma glucose, and concentrations of N(G)-monomethyl-L-arginine (L-NMMA) and N(G), N(G)-dimethyl-L-arginine (ADMA) in endothelial cells, that is, the intima:media ratio became greater as plasma glucose and endothelial L-NMMA and ADMA were increased. Furthermore, endothelial L-NMMA and ADMA were increased in proportion to the increase in plasma glucose. 4. In contrast, there were inverse and significant correlations between cyclic GMP production by carotid artery strips with endothelium and plasma glucose, between cyclic GMP production and endothelial L-NMMA and ADMA, and between the intima:media ratio and cyclic GMP production. 5. Exogenously applied L-NMMA and ADMA inhibited cyclic GMP production in a concentration-dependent manner. IC50 values were determined to be 12.1 microM for the former and 26.2 microM for the latter. The cyclic GMP production was abolished after the deliberate removal of endothelium from the artery strips. 6. These results suggest that the augmentation of intimal hyperplasia with hyperglycaemia is closely related to increased accumulation of L-NMMA and ADMA with hyperglycaemia, which would result in an accelerated reduction in NO production/release by endothelial cells. (+info)Expression of relaxin-like factor is down-regulated in human testicular Leydig cell neoplasia. (8/4620)
In addition to their role in steroidogenesis in the male, testicular Leydig cells constitutively express large amounts of the peptide relaxin-like factor (RLF), also known as Ley-IL. The Leydig cell-derived RLF belongs to the insulin-like superfamily, which also includes relaxin, insulin and the insulin-like growth factors, and within the testis is a specific marker of Leydig cells. Little information is available either on the regulation of gene expression or on the function of this Leydig cell-derived peptide. In the present study we have investigated the expression pattern of human RLF in patients with rare Leydig cell hyperplasia and adenoma. The expression of both mRNA and protein appear to be decreased in hyperplastic Leydig cells, whereas in the Leydig cell adenomas studied, large central areas of the adenoma were devoid of RLF mRNA and protein. Only Leydig cells located at the periphery of the adenoma displayed expression of RLF, with full agreement between in-situ hybridization and immunohistochemistry. It thus appears that the expression of the RLF gene and its products are down-regulated in Leydig cell hyperplasia and adenoma, consistent with a concomitant dedifferentiation of these cells. (+info)There are different types of hyperplasia, depending on the location and cause of the condition. Some examples include:
1. Benign hyperplasia: This type of hyperplasia is non-cancerous and does not spread to other parts of the body. It can occur in various tissues and organs, such as the uterus (fibroids), breast tissue (fibrocystic changes), or prostate gland (benign prostatic hyperplasia).
2. Malignant hyperplasia: This type of hyperplasia is cancerous and can invade nearby tissues and organs, leading to serious health problems. Examples include skin cancer, breast cancer, and colon cancer.
3. Hyperplastic polyps: These are abnormal growths that occur in the gastrointestinal tract and can be precancerous.
4. Adenomatous hyperplasia: This type of hyperplasia is characterized by an increase in the number of glandular cells in a specific organ, such as the colon or breast. It can be a precursor to cancer.
The symptoms of hyperplasia depend on the location and severity of the condition. In general, they may include:
* Enlargement or swelling of the affected tissue or organ
* Pain or discomfort in the affected area
* Abnormal bleeding or discharge
* Changes in bowel or bladder habits
* Unexplained weight loss or gain
Hyperplasia is diagnosed through a combination of physical examination, imaging tests such as ultrasound or MRI, and biopsy. Treatment options depend on the underlying cause and severity of the condition, and may include medication, surgery, or other interventions.
Treatment options include medications such as alpha-blockers and 5-alpha-reductase inhibitors, minimally invasive therapies such as transurethral microwave therapy or laser therapy, and surgical intervention such as a transurethral resection of the prostate (TURP) or robotic-assisted laparoscopic surgery.
There are also lifestyle changes that can help manage Prostatic Hyperplasia, including limiting fluid intake before bedtime, avoiding caffeine and alcohol, and following a healthy diet. It is important to consult with a healthcare professional for proper diagnosis and treatment of this condition.
In simpler terms, Prostatic Hyperplasia is an enlargement of the prostate gland which can cause urinary problems and discomfort. Treatment options include medication, minimally invasive therapies, and surgery, and lifestyle changes can also help manage the condition.
Symptoms of Endometrial Hyperplasia:
The symptoms of endometrial hyperplasia may include:
* Abnormal vaginal bleeding or spotting
* Heavy menstrual periods
* Prolonged menstrual periods
* Painful periods
* Abdominal pain or discomfort
Diagnosis of Endometrial Hyperplasia:
To diagnose endometrial hyperplasia, a doctor may perform the following tests:
* Pelvic examination to check for any abnormalities in the uterus, ovaries, and fallopian tubes.
* Endometrial biopsy to collect a sample of tissue from the endometrium for further examination under a microscope.
* Ultrasound to create images of the uterus and check for any abnormal growths or tumors.
* Hysteroscopy, which is a procedure where a small camera is inserted into the uterus through the cervix to examine the inside of the uterus.
Treatment of Endometrial Hyperplasia:
The treatment of endometrial hyperplasia depends on the severity of the condition and may include:
* Hormonal medications to regulate hormone levels and reduce the growth of the endometrium.
* Endometrial ablation, which is a procedure that destroys the endometrium using heat or cold.
* Hysterectomy, which is the surgical removal of the uterus.
Prevention of Endometrial Hyperplasia:
To prevent endometrial hyperplasia, women can take the following steps:
* Maintain a healthy weight to reduce the risk of hormonal imbalances.
* Exercise regularly to improve overall health and reduce the risk of hormonal imbalances.
* Avoid exposure to endocrine disruptors, such as pesticides and herbicides, which can mimic or interfere with hormones in the body.
* Limit alcohol consumption, as excessive alcohol consumption can increase the risk of hormonal imbalances.
* Eat a balanced diet that is rich in fruits, vegetables, and whole grains, which can help regulate hormone levels.
* Consider taking supplements such as vitamin D and omega-3 fatty acids, which have been shown to have anti-inflammatory effects and may help regulate hormone levels.
It is important for women to talk to their healthcare provider about their individual risk factors for endometrial hyperplasia and any steps they can take to prevent the condition.
There are three main forms of ACH:
1. Classic congenital adrenal hyperplasia (CAH): This is the most common form of ACH, accounting for about 90% of cases. It is caused by mutations in the CYP21 gene, which codes for an enzyme that converts cholesterol into cortisol and aldosterone.
2. Non-classic CAH (NCAH): This form of ACH is less common than classic CAH and is caused by mutations in other genes involved in cortisol and aldosterone production.
3. Mineralocorticoid excess (MOE) or glucocorticoid deficiency (GD): These are rare forms of ACH that are characterized by excessive production of mineralocorticoids (such as aldosterone) or a deficiency of glucocorticoids (such as cortisol).
The symptoms of ACH can vary depending on the specific form of the disorder and the age at which it is diagnosed. In classic CAH, symptoms typically appear in infancy and may include:
* Premature puberty (in girls) or delayed puberty (in boys)
* Abnormal growth patterns
* Distended abdomen
* Fatigue
* Weight gain or obesity
* Easy bruising or bleeding
In NCAH and MOE/GD, symptoms may be less severe or may not appear until later in childhood or adulthood. They may include:
* High blood pressure
* Low blood sugar (hypoglycemia)
* Weight gain or obesity
* Fatigue
* Mood changes
If left untreated, ACH can lead to serious complications, including:
* Adrenal gland insufficiency
* Heart problems
* Bone health problems
* Increased risk of infections
* Mental health issues (such as depression or anxiety)
Treatment for ACH typically involves hormone replacement therapy to restore the balance of hormones in the body. This may involve taking medications such as cortisol, aldosterone, or other hormones to replace those that are deficient or imbalanced. In some cases, surgery may be necessary to remove an adrenal tumor or to correct physical abnormalities.
With proper treatment, many individuals with ACH can lead healthy, active lives. However, it is important for individuals with ACH to work closely with their healthcare providers to manage their condition and prevent complications. This may involve regular check-ups, hormone level monitoring, and lifestyle changes such as a healthy diet and regular exercise.
The condition is characterized by the excessive growth of gum tissue, which can lead to:
1. Redness and swelling of the gums
2. Bleeding while brushing or flossing
3. Bad breath (halitosis)
4. Pocket formation between the teeth and gums
5. Gum recession
6. Tooth loss
Gingival hyperplasia can be treated by addressing the underlying cause, improving oral hygiene, and undergoing scaling and root planing procedures to remove plaque and tartar. In severe cases, surgical intervention may be necessary to remove excess gum tissue and restore the natural contours of the mouth.
It is important for individuals to practice good oral hygiene, including brushing at least twice a day with fluoride toothpaste, flossing daily, and receiving regular dental cleanings to prevent gingival hyperplasia and other gum diseases. Early detection and treatment can help prevent the progression of the condition and restore the health of the teeth and gums.
In thymus hyperplasia, the thymus gland becomes larger than normal and produces more T-cells than needed, which can lead to an imbalance in the immune system. This condition is rare and usually occurs in infants or young children. It can be caused by genetic mutations or viral infections such as HIV.
Symptoms of thymus hyperplasia include fever, fatigue, loss of appetite, and enlargement of the lymph nodes. Treatment for this condition usually involves antiviral medications to reduce the viral load if the condition is caused by HIV, or surgical removal of the enlarged thymus gland if the condition is due to a genetic mutation.
It's important to note that thymus hyperplasia is different from thymoma, which is a type of cancer that affects the thymus gland. Thymoma is rare and usually occurs in adults. In contrast, thymus hyperplasia is more common in children and can be caused by both genetic mutations and viral infections.
The exact cause of FNH is not fully understood, but it is believed to be related to hormonal factors, genetic mutations, or chronic liver injury. The lesion usually grows slowly over time, and it can range in size from a few millimeters to several centimeters in diameter.
The symptoms of FNH are generally non-specific and can include abdominal pain, fatigue, and mild elevations in liver enzymes. However, many patients with FNH may not experience any symptoms at all. The lesion is typically detected incidentally during imaging studies performed for other medical conditions.
The diagnosis of FNH is based on a combination of imaging studies and pathological examination of a liver biopsy specimen. Imaging studies, such as ultrasound, computed tomography (CT), or magnetic resonance imaging (MRI), can help to identify the size and location of the lesion. However, the only way to definitively diagnose FNH is by performing a liver biopsy, which involves inserting a needle into the liver and withdrawing a small sample of tissue for pathological examination.
The treatment of FNH depends on the size and location of the lesion, as well as the patient's overall health status. Small lesions that are less than 3 cm in diameter may not require any treatment, while larger lesions may be treated with liver ablation therapies, such as radiofrequency ablation or laser ablation. In some cases, surgical resection of the affected liver lobe may be necessary.
In summary, focal nodular hyperplasia (FNH) is a benign liver lesion that is characterized by a focal proliferation of hepatocytes and bile ducts within a well-defined nodule. Imaging studies and liver biopsy are essential for diagnosis, and treatment options depend on the size and location of the lesion.
FEH is considered a precancerous condition, as it has the potential to progress to cervical or endometrial cancer if left untreated. However, with early detection and appropriate treatment, the risk of progression to cancer is low.
The symptoms of FEH are not specific and can include vaginal bleeding, abnormal discharge, pain during sex, and pelvic pain. The condition is typically diagnosed through a pap smear or colposcopy, which can identify abnormal cells in the cervix or endometrium.
Treatment for FEH usually involves removal of the affected tissue through a procedure called loop electrosurgical excision procedure (LEEP) or cold knife cone biopsy. In some cases, hysterectomy may be recommended if the condition is severe or persistent.
It is important for women to practice good gynecologic health, including regular pap smears and pelvic exams, to detect any abnormalities early on and prevent the progression of FEH to cancer.
Neointima can be observed in various cardiovascular conditions such as atherosclerosis, stenosis, and graft stenosis. The thickness of the neointima is an important predictor of cardiovascular events such as restenosis after angioplasty or stenting.
Neointima can be characterized using various imaging techniques such as intravascular ultrasound (IVUS) and optical coherence tomography (OCT), which provide detailed information on the morphology and composition of the neointima.
Understanding the mechanisms of neointima formation and its role in cardiovascular disease can help to develop new therapeutic strategies for preventing or treating these conditions.
The exact cause of ALHE is not known, but it is believed to be due to a combination of genetic and environmental factors. The condition typically affects individuals in their teenage years or early adulthood and is more common in women than men.
The symptoms of ALHE can vary depending on the location and severity of the condition, but may include swelling, pain, and redness in the affected limb. Diagnosis is based on a combination of clinical findings, imaging studies (such as ultrasound or CT scan), and biopsy.
Treatment for ALHE typically involves observation and management of symptoms with non-steroidal anti-inflammatory drugs (NSAIDs) or corticosteroids. In some cases, surgical intervention may be necessary to remove affected tissue. The prognosis for ALHE is generally good, but the condition can recur in some individuals.
There is currently no known prevention for ALHE, and the exact cause of the condition is not fully understood. Further research is needed to better understand the etiology and pathophysiology of ALHE and to develop more effective treatment strategies.
Examples of precancerous conditions include:
1. Dysplasia: This is a condition where abnormal cells are present in the tissue, but have not yet invaded surrounding tissues. Dysplasia can be found in organs such as the cervix, colon, and breast.
2. Carcinoma in situ (CIS): This is a condition where cancer cells are present in the tissue, but have not yet invaded surrounding tissues. CIS is often found in organs such as the breast, prostate, and cervix.
3. Atypical hyperplasia: This is a condition where abnormal cells are present in the tissue, but they are not yet cancerous. Atypical hyperplasia can be found in organs such as the breast and uterus.
4. Lobular carcinoma in situ (LCIS): This is a condition where cancer cells are present in the milk-producing glands of the breasts, but have not yet invaded surrounding tissues. LCIS is often found in both breasts and can increase the risk of developing breast cancer.
5. Adenomas: These are small growths on the surface of the colon that can become malignant over time if left untreated.
6. Leukoplakia: This is a condition where thick, white patches develop on the tongue or inside the mouth. Leukoplakia can be a precancerous condition and may increase the risk of developing oral cancer.
7. Oral subsquamous carcinoma: This is a type of precancerous lesion that develops in the mouth and can progress to squamous cell carcinoma if left untreated.
8. Cervical intraepithelial neoplasia (CIN): This is a condition where abnormal cells are present on the surface of the cervix, but have not yet invaded surrounding tissues. CIN can progress to cancer over time if left untreated.
9. Vulvar intraepithelial neoplasia (VIN): This is a condition where abnormal cells are present on the vulva, but have not yet invaded surrounding tissues. VIN can progress to cancer over time if left untreated.
10. Penile intraepithelial neoplasia (PIN): This is a condition where abnormal cells are present on the penis, but have not yet invaded surrounding tissues. PIN can progress to cancer over time if left untreated.
It is important to note that not all precancerous conditions will develop into cancer, and some may resolve on their own without treatment. However, it is important to follow up with a healthcare provider to monitor any changes and determine the best course of treatment.
There are several different types of pseudolymphoma, including:
1. Cutaneous pseudolymphoma: This type of pseudolymphoma affects the skin and can appear as a rash or lump.
2. Nodular pseudolymphoma: This type of pseudolymphoma affects the lymph nodes and can cause swelling in the neck, armpits, or groin.
3. Extranodal pseudolymphoma: This type of pseudolymphoma can occur outside of the lymph nodes and can affect other organs, such as the spleen or liver.
4. Lymphomatoid papulosis: This is a rare type of pseudolymphoma that can cause recurring episodes of swelling in the lymph nodes or skin.
The exact cause of pseudolymphoma is not known, but it is thought to be related to an abnormal response of the immune system. Treatment for pseudolymphoma typically involves surgery to remove the affected tissue, as well as radiation therapy and chemotherapy to kill any remaining cancer cells. In some cases, the condition may go into remission on its own without treatment.
Endometrial neoplasms are abnormal growths or tumors that develop in the lining of the uterus, known as the endometrium. These growths can be benign (non-cancerous) or malignant (cancerous). The most common type of endometrial neoplasm is endometrial hyperplasia, which is a condition where the endometrium grows too thick and can become cancerous if left untreated. Other types of endometrial neoplasms include endometrial adenocarcinoma, which is the most common type of uterine cancer, and endometrial sarcoma, which is a rare type of uterine cancer that develops in the muscle or connective tissue of the uterus.
Endometrial neoplasms can be caused by a variety of factors, including hormonal imbalances, genetic mutations, and exposure to certain chemicals or radiation. Risk factors for developing endometrial neoplasms include obesity, early onset of menstruation, late onset of menopause, never being pregnant or having few or no full-term pregnancies, and taking hormone replacement therapy or other medications that can increase estrogen levels.
Symptoms of endometrial neoplasms can include abnormal vaginal bleeding, painful urination, and pelvic pain or discomfort. Treatment for endometrial neoplasms depends on the type and stage of the condition, and may involve surgery, radiation therapy, chemotherapy, or hormone therapy. In some cases, a hysterectomy (removal of the uterus) may be necessary.
In summary, endometrial neoplasms are abnormal growths that can develop in the lining of the uterus and can be either benign or malignant. They can be caused by a variety of factors and can cause symptoms such as abnormal bleeding and pelvic pain. Treatment depends on the type and stage of the condition, and may involve surgery, radiation therapy, chemotherapy, or hormone therapy.
There are several types of carotid artery injuries, including:
1. Carotid artery dissection: This is a tear in the inner lining of the artery that can lead to bleeding and inflammation.
2. Carotid artery thrombosis: This is the formation of a blood clot within the artery that can block blood flow to the brain.
3. Carotid artery occlusion: This is the complete blockage of the artery, which can cause a stroke or transient ischemic attack (TIA).
4. Carotid artery injury due to trauma: This type of injury can occur as a result of a blow to the neck or head.
5. Carotid artery injury due to surgery: This type of injury can occur during surgical procedures that involve the carotid arteries, such as endarterectomy or stenting.
The symptoms of carotid artery injuries can vary depending on the severity of the injury and the location of the damage. Some common symptoms include:
* Sudden weakness or numbness in the face, arm, or leg
* Sudden confusion or trouble speaking
* Sudden vision loss or double vision
* Sudden difficulty walking or maintaining balance
* Sudden severe headache
The diagnosis of carotid artery injuries is typically made using imaging tests such as ultrasound, computed tomography (CT) scans, or magnetic resonance imaging (MRI). Treatment options for carotid artery injuries depend on the severity and location of the injury, and may include medications, endovascular procedures, or surgery.
Prevention of carotid artery injuries is key to reducing the risk of complications. This can be achieved through:
* Maintaining a healthy lifestyle, including regular exercise and a balanced diet
* Avoiding smoking and limiting alcohol consumption
* Managing underlying medical conditions such as high blood pressure or diabetes
* Properly managing medications that may increase the risk of bleeding or injury
* Using appropriate precautions during surgical procedures, such as using sterile equipment and monitoring for signs of bleeding or injury.
In conclusion, carotid artery injuries can have serious consequences if left untreated. It is important to be aware of the causes, symptoms, diagnosis, and treatment options for these injuries in order to provide appropriate care and prevent complications. Proper precautions during surgical procedures and a healthy lifestyle can also help reduce the risk of carotid artery injuries.
Adenomas are caused by genetic mutations that occur in the DNA of the affected cells. These mutations can be inherited or acquired through exposure to environmental factors such as tobacco smoke, radiation, or certain chemicals.
The symptoms of an adenoma can vary depending on its location and size. In general, they may include abdominal pain, bleeding, or changes in bowel movements. If the adenoma becomes large enough, it can obstruct the normal functioning of the affected organ or cause a blockage that can lead to severe health complications.
Adenomas are usually diagnosed through endoscopy, which involves inserting a flexible tube with a camera into the affected organ to visualize the inside. Biopsies may also be taken to confirm the presence of cancerous cells.
Treatment for adenomas depends on their size, location, and severity. Small, non-pedunculated adenomas can often be removed during endoscopy through a procedure called endoscopic mucosal resection (EMR). Larger adenomas may require surgical resection, and in some cases, chemotherapy or radiation therapy may also be necessary.
In summary, adenoma is a type of benign tumor that can occur in glandular tissue throughout the body. While they are not cancerous, they have the potential to become malignant over time if left untreated. Therefore, it is important to seek medical attention if symptoms persist or worsen over time. Early detection and treatment can help prevent complications and improve outcomes for patients with adenomas.
Graft occlusion can occur due to a variety of factors, including:
1. Blood clots forming within the graft
2. Inflammation or infection within the graft
3. Narrowing or stenosis of the graft
4. Disruption of the graft material
5. Poor blood flow through the graft
The signs and symptoms of vascular graft occlusion can vary depending on the location and severity of the blockage. They may include:
1. Pain or tenderness in the affected limb
2. Swelling or redness in the affected limb
3. Weakness or numbness in the affected limb
4. Difficulty walking or moving the affected limb
5. Coolness or discoloration of the skin in the affected limb
If you experience any of these symptoms, it is important to seek medical attention as soon as possible. A healthcare professional can diagnose vascular graft occlusion using imaging tests such as ultrasound, angiography, or MRI. Treatment options for vascular graft occlusion may include:
1. Medications to dissolve blood clots or reduce inflammation
2. Surgical intervention to repair or replace the graft
3. Balloon angioplasty or stenting to open up the blocked graft
4. Hyperbaric oxygen therapy to improve blood flow and promote healing.
Preventive measures to reduce the risk of vascular graft occlusion include:
1. Proper wound care and infection prevention after surgery
2. Regular follow-up appointments with your healthcare provider
3. Avoiding smoking and other cardiovascular risk factors
4. Taking medications as directed by your healthcare provider to prevent blood clots and inflammation.
It is important to note that vascular graft occlusion can be a serious complication after surgery, but with prompt medical attention and appropriate treatment, the outcome can be improved.
In this condition, the lymph nodes become larger than normal and can be found in various parts of the body, such as the neck, underarm, or groin. The enlarged lymph nodes may or may not cause any symptoms, but they can be detected through a physical examination or imaging tests such as CT scans or MRI scans.
The exact cause of giant lymph node hyperplasia is not known, but it is believed to be related to an abnormal immune response or exposure to certain infections or chemicals. Treatment options for the condition include surgical removal of the enlarged lymph nodes, radiation therapy, or chemotherapy, depending on the severity and location of the growths.
1) They share similarities with humans: Many animal species share similar biological and physiological characteristics with humans, making them useful for studying human diseases. For example, mice and rats are often used to study diseases such as diabetes, heart disease, and cancer because they have similar metabolic and cardiovascular systems to humans.
2) They can be genetically manipulated: Animal disease models can be genetically engineered to develop specific diseases or to model human genetic disorders. This allows researchers to study the progression of the disease and test potential treatments in a controlled environment.
3) They can be used to test drugs and therapies: Before new drugs or therapies are tested in humans, they are often first tested in animal models of disease. This allows researchers to assess the safety and efficacy of the treatment before moving on to human clinical trials.
4) They can provide insights into disease mechanisms: Studying disease models in animals can provide valuable insights into the underlying mechanisms of a particular disease. This information can then be used to develop new treatments or improve existing ones.
5) Reduces the need for human testing: Using animal disease models reduces the need for human testing, which can be time-consuming, expensive, and ethically challenging. However, it is important to note that animal models are not perfect substitutes for human subjects, and results obtained from animal studies may not always translate to humans.
6) They can be used to study infectious diseases: Animal disease models can be used to study infectious diseases such as HIV, TB, and malaria. These models allow researchers to understand how the disease is transmitted, how it progresses, and how it responds to treatment.
7) They can be used to study complex diseases: Animal disease models can be used to study complex diseases such as cancer, diabetes, and heart disease. These models allow researchers to understand the underlying mechanisms of the disease and test potential treatments.
8) They are cost-effective: Animal disease models are often less expensive than human clinical trials, making them a cost-effective way to conduct research.
9) They can be used to study drug delivery: Animal disease models can be used to study drug delivery and pharmacokinetics, which is important for developing new drugs and drug delivery systems.
10) They can be used to study aging: Animal disease models can be used to study the aging process and age-related diseases such as Alzheimer's and Parkinson's. This allows researchers to understand how aging contributes to disease and develop potential treatments.
Some common types of parathyroid diseases include:
1. Hyperparathyroidism: This is a condition in which the parathyroid glands produce too much PTH, leading to high levels of calcium in the blood. It can be caused by a benign tumor or by genetic mutations.
2. Hypoparathyroidism: This is a condition in which the parathyroid glands do not produce enough PTH, leading to low levels of calcium in the blood. It can be caused by autoimmune disorders, radiation therapy, or surgical removal of the parathyroid glands.
3. Parathyroid cancer: This is a rare type of cancer that affects the parathyroid glands. It can cause symptoms such as neck swelling, hoarseness, and difficulty swallowing.
4. Familial isolated hyperparathyroidism (FIH): This is a genetic condition that causes benign tumors to grow on one or more of the parathyroid glands, leading to high levels of calcium in the blood.
5. Parathyroid hormone-secreting pancreatic neuroendocrine tumors (PTH-Secreting PNETs): These are rare tumors that occur in the pancreas and produce excessive amounts of PTH, leading to high levels of calcium in the blood.
Treatment options for parathyroid diseases depend on the specific type and severity of the condition. Surgery is often necessary to remove affected glands or tumors, and medications may be used to manage symptoms such as high blood pressure and kidney stones. In some cases, hormone replacement therapy may be needed to replace missing PTH.
Adrenocortical hyperfunction can be caused by a variety of factors, including:
1. Cushing's syndrome: This is a rare hormonal disorder caused by excessive production of cortisol by the adrenal glands. It can be caused by a benign tumor in the pituitary gland or an adrenal gland, or by taking too much corticosteroid medication.
2. Adrenocortical carcinoma: This is a rare and aggressive type of cancer that affects the adrenal glands. It can cause excessive production of cortisol and other hormones.
3. Familial Cushing's syndrome: This is a genetic disorder that causes excessive production of cortisol by the adrenal glands.
4. Glucocorticoid-remediable aldosteronism (GRA): This is a rare genetic disorder that affects the production of hormones by the adrenal glands, leading to excessive production of cortisol and aldosterone.
5. Licorice ingestion: Consuming large amounts of licorice can cause an increase in the production of cortisol and other hormones by the adrenal glands.
6. Primary aldosteronism (PA): This is a group of rare genetic disorders that affect the production of hormones by the adrenal glands, leading to excessive production of aldosterone and cortisol.
7. Secondary adrenocortical hyperfunction: This can occur due to various conditions such as thyroid disorders, pituitary tumors, or other endocrine disorders that affect the regulation of hormone production by the adrenal glands.
Symptoms of adrenocortical hyperfunction may include:
1. Weight gain and central obesity
2. Increased appetite and food cravings
3. Fatigue and weakness
4. Mood changes, such as anxiety or depression
5. Insomnia and sleep disturbances
6. High blood pressure and cardiovascular risk factors
7. Easy bruising and poor wound healing
8. Muscle weakness and fatigue
9. Thinning of the skin and bones (osteoporosis)
10. Increased risk of infections and decreased immune function.
If you suspect that you or someone you know may have adrenocortical hyperfunction, it is essential to consult with a healthcare professional for proper diagnosis and treatment. A doctor may perform several tests, including:
1. Blood tests to measure hormone levels in the body, such as cortisol and aldosterone.
2. Saliva tests to measure cortisol levels throughout the day.
3. Urine tests to measure cortisol levels over a 24-hour period.
4. Imaging tests, such as CT scans or MRI scans, to examine the adrenal glands and look for any signs of tumors or other abnormalities.
5. Other tests to assess the body's response to stress, such as a corticotropin (ACTH) stimulation test.
Treatment options for adrenocortical hyperfunction depend on the underlying cause of the condition and may include:
1. Medications to reduce hormone production in the adrenal glands, such as metyrapone or ketoconazole.
2. Surgery to remove any tumors or cysts in the adrenal glands.
3. Radiation therapy to shrink tumors and reduce hormone production.
4. Lifestyle changes, such as weight loss, stress management techniques, and regular exercise.
5. Monitoring of hormone levels and other health markers to ensure that the condition is under control.
There are several types of hypertrophy, including:
1. Muscle hypertrophy: The enlargement of muscle fibers due to increased protein synthesis and cell growth, often seen in individuals who engage in resistance training exercises.
2. Cardiac hypertrophy: The enlargement of the heart due to an increase in cardiac workload, often seen in individuals with high blood pressure or other cardiovascular conditions.
3. Adipose tissue hypertrophy: The excessive growth of fat cells, often seen in individuals who are obese or have insulin resistance.
4. Neurological hypertrophy: The enlargement of neural structures such as brain or spinal cord due to an increase in the number of neurons or glial cells, often seen in individuals with neurodegenerative diseases such as Alzheimer's or Parkinson's.
5. Hepatic hypertrophy: The enlargement of the liver due to an increase in the number of liver cells, often seen in individuals with liver disease or cirrhosis.
6. Renal hypertrophy: The enlargement of the kidneys due to an increase in blood flow and filtration, often seen in individuals with kidney disease or hypertension.
7. Ovarian hypertrophy: The enlargement of the ovaries due to an increase in the number of follicles or hormonal imbalances, often seen in individuals with polycystic ovary syndrome (PCOS).
Hypertrophy can be diagnosed through various medical tests such as imaging studies (e.g., CT scans, MRI), biopsies, and blood tests. Treatment options for hypertrophy depend on the underlying cause and may include medications, lifestyle changes, and surgery.
In conclusion, hypertrophy is a growth or enlargement of cells, tissues, or organs in response to an excessive stimulus. It can occur in various parts of the body, including the brain, liver, kidneys, heart, muscles, and ovaries. Understanding the underlying causes and diagnosis of hypertrophy is crucial for effective treatment and management of related health conditions.
Papillomas can occur anywhere on the body, but they are most commonly found on the face, neck, and scalp. They may appear as small bumps or growths that look like a wart. In some cases, papillomas may be associated with human papillomavirus (HPV) infection.
Papillomas are typically diagnosed through a physical examination of the affected area. In some cases, a biopsy may be performed to confirm the diagnosis and rule out other potential causes. Treatment for papillomas usually involves removal of the growth through a minor surgical procedure or cryotherapy (freezing).
Papillomas are not cancerous and do not typically pose any long-term health risks. However, they may be unsightly and can cause psychological distress for some people. In these cases, treatment may be sought for cosmetic reasons. It is important to note that papillomas should not be confused with squamous cell carcinoma, a type of skin cancer that can resemble a papilloma in appearance but has the potential to be more aggressive and harmful.
Types of Parathyroid Neoplasms: There are several types of parathyroid neoplasms, including:
1. Adenoma: A benign tumor that is the most common type of parathyroid neoplasm. It usually causes hyperparathyroidism, a condition characterized by high levels of calcium in the blood.
2. Hyperplasia: A condition where the parathyroid glands become enlarged and produce excessive amounts of parathyroid hormone, leading to hyperparathyroidism.
3. Carcinoma: A malignant tumor that is rare and usually occurs in patients with a history of radiation exposure or familial adenomatous polyposis (FAP).
Symptoms of Parathyroid Neoplasms: The symptoms of parathyroid neoplasms can vary depending on the type and size of the tumor. Some common symptoms include:
1. Hyperparathyroidism: High levels of calcium in the blood, which can lead to symptoms such as fatigue, nausea, vomiting, and weakness.
2. Enlarged thyroid gland: A swelling in the neck due to an enlarged thyroid gland, which can cause difficulty swallowing or breathing.
3. Pain in the neck or throat: A painful lump in the neck or throat that can be caused by a tumor pressing on nearby structures.
4. Fever: An elevated body temperature that can occur if the tumor becomes infected or inflamed.
5. Weight loss: Unexplained weight loss, which can occur if the tumor is secreting excessive amounts of parathyroid hormone.
Diagnosis of Parathyroid Neoplasms: The diagnosis of parathyroid neoplasms typically involves a combination of imaging studies and laboratory tests. Some common diagnostic procedures include:
1. Ultrasound: A non-invasive imaging technique that uses high-frequency sound waves to produce images of the thyroid gland and any tumors present.
2. Thyroid scan: A nuclear medicine test that involves injecting a small amount of radioactive material into the bloodstream to visualize the thyroid gland and any tumors present.
3. Calcium levels: Blood tests to measure calcium levels, which can be elevated in hyperparathyroidism.
4. Parathyroid hormone (PTH) level: A blood test to measure PTH levels, which can be elevated in hyperparathyroidism.
5. Biopsy: A procedure that involves removing a small sample of tissue from the thyroid gland and examining it under a microscope for cancer cells.
Treatment of Parathyroid Neoplasms: The treatment of parathyroid neoplasms depends on the type and size of the tumor, as well as the severity of hyperparathyroidism. Some common treatments include:
1. Surgery: The primary treatment for parathyroid neoplastic diseases is surgical removal of the affected parathyroid gland(s).
2. Radioactive iodine ablation: A therapy that involves taking a small dose of radioactive iodine to destroy any remaining thyroid tissue that may be producing excessive amounts of thyroid hormones.
3. Thyroid hormone medications: Medications that are used to control hyperthyroidism and hypothyroidism.
4. Calcium and vitamin D supplements: Medications that are used to treat hypocalcemia and vitamin D deficiency.
5. Monitoring: Regular monitoring of calcium levels, PTH levels, and symptoms is important to ensure that the treatment is effective and to detect any recurrences or complications.
Prognosis: The prognosis for patients with parathyroid neoplasms depends on the type and size of the tumor, as well as the severity of hyperparathyroidism. In general, the prognosis is good for patients who undergo surgical removal of the affected gland(s), but it may be poorer for those with more advanced or invasive tumors.
Complications: Complications of parathyroid neoplasms include:
1. Hyperparathyroidism: Excessive production of PTH can lead to hyperthyroidism, hypocalcemia, and other complications.
2. Recurrence: There is a risk of recurrence after surgical removal of the affected gland(s).
3. Spread of disease: In rare cases, parathyroid tumors can spread to other parts of the body (such as the lymph nodes or bones) and cause metastatic disease.
4. Hypoparathyroidism: Removal of all four parathyroid glands can lead to hypoparathyroidism, which can be life-threatening if not treated promptly.
5. Pancreatitis: Some studies have suggested that there may be an increased risk of pancreatitis in patients with parathyroid neoplasms.
Cushing syndrome is a rare hormonal disorder that occurs when the body produces too much cortisol, a steroid hormone produced by the adrenal gland. It can be caused by a variety of factors, including tumors, infections, and genetic conditions.
The symptoms of Cushing syndrome can vary depending on the cause and severity of the condition, but may include:
* Weight gain, particularly in the abdomen, face, and neck
* Fatigue and muscle weakness
* Poor sleep
* Mood changes, such as anxiety, depression, and irritability
* High blood pressure
* Easy bruising and thinning skin
* Osteoporosis or osteopenia
* Increased risk of infections
* Menstrual irregularities in women
* Hirsutism (excessive hair growth) in women
* Erectile dysfunction in men
Cushing syndrome can be difficult to diagnose, as the symptoms can be similar to other conditions. A healthcare provider will typically begin by taking a detailed medical history and performing a physical exam. They may also order several tests, including:
* Blood tests to measure cortisol levels and look for other hormonal imbalances
* Urine tests to check for abnormal steroid metabolites
* Imaging studies, such as CT or MRI scans, to look for tumors or other structural abnormalities
* Salivary cortisol testing to measure cortisol levels throughout the day
Treatment for Cushing syndrome depends on the underlying cause of the condition. In some cases, medication may be prescribed to reduce cortisol production or to treat symptoms such as high blood pressure or mood changes. Surgery may be necessary to remove a tumor or other structural abnormality. In addition, lifestyle changes such as diet and exercise may be recommended to help manage the condition.
It is important for individuals with Cushing syndrome to work closely with their healthcare provider to develop a treatment plan that is tailored to their specific needs and circumstances. With appropriate treatment, many people with Cushing syndrome can experience significant improvement in their symptoms and quality of life.
Some common types of breast diseases include:
1. Fibrocystic breast disease: A condition characterized by the formation of small lumps or cysts in the breast tissue, often accompanied by breast pain.
2. Fibroadenoma: A benign tumor that is made up of glandular and fibrous tissue, typically found in young women.
3. Ductal carcinoma in situ (DCIS): A type of cancer that originates in the milk ducts and has not spread to other parts of the breast or body.
4. Invasive ductal carcinoma (IDC): The most common type of breast cancer, which starts in the milk ducts but has invaded the surrounding tissue.
5. Invasive lobular carcinoma (ILC): A type of breast cancer that starts in the milk-producing glands (lobules) and has invaded the surrounding tissue.
6. Breast abscess: A collection of pus in the breast tissue, often caused by an infection.
7. Mastitis: An inflammation of the breast tissue, usually caused by a bacterial infection.
8. Breast cancer recurrence: Cancer that has returned after previous treatment.
Early detection and diagnosis of breast diseases are crucial for effective treatment and improved outcomes. Screening tests such as mammography, ultrasound, and biopsy can help identify breast diseases at an early stage, when they are more treatable. Treatment options for breast diseases depend on the type and severity of the condition, and may include surgery, chemotherapy, radiation therapy, or hormone therapy.
Malignant prostatic neoplasms are cancerous tumors that can be aggressive and spread to other parts of the body (metastasize). The most common type of malignant prostatic neoplasm is adenocarcinoma of the prostate, which accounts for approximately 95% of all prostate cancers. Other types of malignant prostatic neoplasms include sarcomas and small cell carcinomas.
Prostatic neoplasms can be diagnosed through a variety of tests such as digital rectal examination (DRE), prostate-specific antigen (PSA) test, imaging studies (ultrasound, CT scan or MRI), and biopsy. Treatment options for prostatic neoplasms depend on the type, stage, and grade of the tumor, as well as the patient's age and overall health. Treatment options can include active surveillance, surgery (robotic-assisted laparoscopic prostatectomy or open prostatectomy), radiation therapy (external beam radiation therapy or brachytherapy), and hormone therapy.
In summary, Prostatic Neoplasms are tumors that occur in the prostate gland, which can be benign or malignant. The most common types of malignant prostatic neoplasms are adenocarcinoma of the prostate, and other types include sarcomas and small cell carcinomas. Diagnosis is done through a variety of tests, and treatment options depend on the type, stage, and grade of the tumor, as well as the patient's age and overall health.
Some common types of adrenal gland diseases include:
1. Cushing's syndrome: A hormonal disorder caused by excessive production of cortisol, a hormone produced by the adrenal glands. This can be caused by a tumor on one of the adrenal glands or by taking too much corticosteroid medication.
2. Addison's disease: A rare disorder caused by the destruction of the adrenal glands, typically due to an autoimmune response. This results in a deficiency of cortisol and aldosterone hormones, leading to symptoms such as fatigue, weight loss, and skin changes.
3. Adrenocortical carcinoma: A rare type of cancer that affects the adrenal glands. This can cause symptoms such as weight gain, skin changes, and abdominal pain.
4. Pheochromocytoma: A rare type of tumor that develops on one of the adrenal glands, typically causing high blood pressure and other symptoms due to excessive production of hormones such as epinephrine and norepinephrine.
5. Adrenal insufficiency: A condition in which the adrenal glands do not produce enough cortisol and aldosterone hormones, often caused by a autoimmune response or a viral infection. This can lead to symptoms such as fatigue, weight loss, and skin changes.
6. Primary aldosteronism: A condition in which the adrenal glands produce too much aldosterone hormone, leading to high blood pressure and other symptoms.
7. Adrenal incidentalomas: Tumors that are found on the adrenal glands, but do not produce excessive hormones or cause symptoms. These tumors can be benign or malignant.
8. Adrenal metastases: Tumors that have spread to the adrenal glands from another part of the body, often causing symptoms such as high blood pressure and abdominal pain.
9. Adrenal cysts: Fluid-filled sacs that form on the adrenal glands, which can cause symptoms such as abdominal pain and weight loss.
10. Adrenal hemorrhage: Bleeding in the adrenal glands, often caused by trauma or a blood clotting disorder. This can lead to symptoms such as severe abdominal pain and shock.
It is important to note that this list is not exhaustive and there may be other rare conditions that affect the adrenal glands not included here. If you suspect you have any of these conditions, it is important to seek medical attention from a qualified healthcare professional for proper diagnosis and treatment.
Also known as CIS.
There are two main types of hyperparathyroidism: primary and secondary. Primary hyperparathyroidism is caused by a benign tumor in one of the parathyroid glands, while secondary hyperparathyroidism is caused by another condition that leads to overproduction of PTH, such as kidney disease or vitamin D deficiency.
Symptoms of hyperparathyroidism can include:
* High blood calcium levels
* Bone loss or osteoporosis
* Kidney stones
* Pancreatitis (inflammation of the pancreas)
* Hyperthyroidism (an overactive thyroid gland)
* Fatigue
* Weakness
* Nausea and vomiting
* Abdominal pain
* Headaches
Treatment for hyperparathyroidism usually involves surgery to remove the affected parathyroid gland or glands. In some cases, medications may be used to manage symptoms before surgery. It is important for individuals with hyperparathyroidism to receive prompt medical attention, as untreated hyperparathyroidism can lead to serious complications such as heart disease and kidney failure.
Adenomas, liver cell are relatively rare and account for only 1-3% of all primary liver tumors. They tend to affect middle-aged adults, and the exact cause is not known. However, certain factors such as cirrhosis, hepatitis B and C, and exposure to certain chemicals have been linked to an increased risk of developing an adenoma.
The diagnosis of an adenoma, liver cell is based on a combination of imaging studies such as ultrasound, computed tomography (CT), and magnetic resonance imaging (MRI) and a biopsy to confirm the presence of malignant cells. Treatment usually involves surgical removal of the tumor, and in some cases, embolization may be used to shrink the tumor before surgery.
It is important to note that not all liver cell adenomas are benign, and some may be premalignant or even malignant. Therefore, it is important to follow up with a healthcare professional regularly after diagnosis to monitor for any changes in the tumor.
It is important to note that while fibrocystic breast disease can be uncomfortable and may resemble cancerous changes on a mammogram, it is not cancer and does not increase the risk of developing breast cancer. However, due to its similar appearance on imaging studies, it is important to have any new or persistent changes evaluated by a healthcare provider to rule out other conditions such as cancer.
Fibrocystic breast disease can be diagnosed through a combination of physical examination, mammography, and ultrasound. Treatment for the condition typically involves managing symptoms with pain medication, anti-inflammatory medications, or hormonal therapy. In some cases, surgery may be necessary to remove cysts or scar tissue that are causing discomfort or other symptoms.
Fibrocystic breast disease is more common in women between the ages of 20 and 50, and it can affect one or both breasts. While there is no specific cause for the condition, hormonal changes, genetics, and other factors may play a role in its development.
Breast self-awareness and regular screening are important for early detection and management of fibrocystic breast disease. Women with this condition should talk to their healthcare provider about their individual risk factors, symptoms, and the best course of treatment.
Types of Adrenal Cortex Neoplasms:
1. Adrenocortical carcinoma (ACC): A rare and aggressive malignant tumor that originates in the adrenal cortex. It is often associated with virilization (excessive masculinization) in women.
2. Adrenocortical adenoma (ACA): A benign tumor that originates in the adrenal cortex. It is less common than ACC and may not cause any symptoms.
3. Pheochromocytoma: A rare tumor that originates in the adrenal medulla, which is the inner part of the adrenal gland. It can secrete excessive amounts of hormones that regulate blood pressure and heart rate.
4. Paraganglioma: A rare tumor that originates in the paraganglia, which are clusters of cells located near the adrenal glands. These tumors can produce excessive amounts of hormones and cause similar symptoms as pheochromocytoma.
Symptoms of Adrenal Cortex Neoplasms:
1. Virilization (excessive masculinization) in women, such as deepening of the voice, excessive body hair growth, and clitoral enlargement.
2. Headache, fatigue, and weight gain due to excessive production of steroid hormones.
3. High blood pressure and heart rate due to excessive production of catecholamines (hormones that regulate blood pressure and heart rate).
4. Abdominal pain, nausea, and vomiting due to the tumor's size and location.
Diagnosis of Adrenal Cortex Neoplasms:
1. Imaging tests such as CT scans or MRI to visualize the tumor and determine its size and location.
2. Laboratory tests to measure hormone levels in the blood, including cortisol, aldosterone, and catecholamines.
3. Biopsy to obtain a tissue sample for further examination under a microscope.
Treatment of Adrenal Cortex Neoplasms:
1. Surgery to remove the tumor, which is usually curative.
2. Medications to control symptoms such as high blood pressure and hormone levels.
3. Radiation therapy may be used in cases where surgery is not feasible or if there is a risk of recurrence.
Prognosis of Adrenal Cortex Neoplasms:
The prognosis for adrenal cortex neoplasms depends on the type and size of the tumor, as well as the extent of hormone production. In general, the prognosis is good for patients with benign tumors that are removed surgically. However, malignant tumors can have a poorer prognosis and may require additional treatments such as radiation therapy or chemotherapy.
Prevention of Adrenal Cortex Neoplasms:
There is no known prevention for adrenal cortex neoplasms, but early detection and treatment can improve outcomes. Regular monitoring of hormone levels and imaging tests can help detect tumors at an early stage.
Lifestyle Changes:
1. Reduce stress: High levels of cortisol can be caused by stress, so finding ways to manage stress can help prevent adrenal cortex neoplasms.
2. Maintain a healthy diet: Eating a balanced diet that includes plenty of fruits, vegetables, and whole grains can help support overall health and well-being.
3. Exercise regularly: Regular physical activity can help reduce stress and improve overall health.
4. Get enough sleep: Aim for 7-8 hours of sleep per night to help regulate hormone levels.
5. Limit caffeine and alcohol: Both substances can disrupt hormone levels and contribute to the development of adrenal cortex neoplasms.
Examples and Observations:
1. Gastric metaplasia: This is a condition where the stomach lining is replaced by cells that are similar to those found in the esophagus. This can occur as a result of chronic acid reflux, leading to an increased risk of developing esophageal cancer.
2. Bronchial metaplasia: This is a condition where the airways in the lungs are replaced by cells that are similar to those found in the trachea. This can occur as a result of chronic inflammation, leading to an increased risk of developing lung cancer.
3. Pancreatic metaplasia: This is a condition where the pancreas is replaced by cells that are similar to those found in the ducts of the pancreas. This can occur as a result of chronic inflammation, leading to an increased risk of developing pancreatic cancer.
4. Breast metaplasia: This is a condition where the breast tissue is replaced by cells that are similar to those found in the salivary glands. This can occur as a result of chronic inflammation, leading to an increased risk of developing salivary gland cancer.
Etiology and Pathophysiology:
Metaplasia is thought to be caused by chronic inflammation, which can lead to the replacement of one type of cell or tissue with another. This can occur as a result of a variety of factors, including infection, injury, or exposure to carcinogens. Once the metaplastic changes have occurred, there is an increased risk of developing cancer if the underlying cause is not addressed.
Clinical Presentation:
Patients with metaplasia may present with a variety of symptoms, depending on the location and extent of the condition. These can include pain, difficulty swallowing or breathing, coughing up blood, and weight loss. In some cases, patients may be asymptomatic and the condition may be detected incidentally during diagnostic testing for another condition.
Diagnosis:
The diagnosis of metaplasia is typically made based on a combination of clinical findings, radiologic imaging (such as CT scans or endoscopies), and histopathological examination of biopsy specimens. Imaging studies can help to identify the location and extent of the metaplastic changes, while histopathology can confirm the presence of the metaplastic cells and rule out other potential diagnoses.
Treatment:
Treatment for metaplasia depends on the underlying cause and the severity of the condition. In some cases, treatment may involve addressing the underlying cause, such as removing a tumor or treating an infection. In other cases, treatment may be directed at managing symptoms and preventing complications. This can include medications to reduce inflammation and pain, as well as surgery to remove affected tissue.
Prognosis:
The prognosis for metaplasia varies depending on the underlying cause and the severity of the condition. In general, the prognosis is good for patients with benign metaplastic changes, while those with malignant changes may have a poorer prognosis if the cancer is not treated promptly and effectively.
Complications:
Metaplasia can lead to a number of complications, including:
1. Cancer: Metaplastic changes can sometimes progress to cancer, which can be life-threatening.
2. Obstruction: The growth of metaplastic cells can block the normal functioning of the organ or gland, leading to obstruction and potentially life-threatening complications.
3. Inflammation: Metaplasia can lead to chronic inflammation, which can cause scarring and further damage to the affected tissue.
4. Bleeding: Metaplastic changes can increase the risk of bleeding, particularly if they occur in the digestive tract or other organs.
Adenomatosis pulmonar is a benign condition that develops when gland-like structures within the lungs (called bronchiolar glands) become enlarged and produce mucus. This can lead to chronic coughing, wheezing, shortness of breath, and chest tightness.
Adenomatosis Pulmonar Symptoms
Symptoms of adenomatosis pulmonar may include:
* Chronic cough that may be worse at night or with exertion
* Wheezing or whistling sounds when breathing out (wheezing)
* Shortness of breath or chest tightness during exercise or activity
* Recurrent lung infections
* Coughing up mucus or phlegm
Adenomatosis Pulmonar Causes
The exact cause of adenomatosis pulmonar is not known, but it is believed to be related to an imbalance of hormones that regulate the growth of bronchiolar glands. Other factors that may contribute to the development of this condition include:
* Allergies
* Asthma
* Chronic respiratory infections
* Exposure to pollutants or irritants
* Hormonal changes during pregnancy or menopause
Adenomatosis Pulmonar Diagnosis
To diagnose adenomatosis pulmonar, a healthcare provider may use the following tests:
* Physical examination and medical history
* Chest X-ray or computed tomography (CT) scan to visualize the lungs and identify any abnormalities
* Pulmonary function tests (PFTs) to assess lung function and breathing capacity
* Bronchoscopy to examine the inside of the airways and collect a tissue sample for biopsy
Adenomatosis Pulmonar Treatment
There is no cure for adenomatosis pulmonar, but treatment can help manage symptoms and slow the progression of the condition. Treatment options may include:
* Bronchodilators to relax airway muscles and improve breathing
* Inhaled corticosteroids to reduce inflammation
* Anti-inflammatory medications to reduce swelling and pain
* Antibiotics to treat any underlying infections
* Oxygen therapy to help improve oxygen levels in the blood
* Pulmonary rehabilitation to improve lung function and overall health
In severe cases, surgery may be necessary to remove damaged tissue or repair damaged airways. Lung transplantation may also be considered in severe cases where other treatments have failed.
Adenomatosis Pulmonar Prognosis
The prognosis for adenomatosis pulmonar is generally poor, with a five-year survival rate of less than 50%. The condition can progress slowly over many years, and the symptoms can be managed with treatment. However, the disease can be unpredictable, and exacerbations can occur at any time. With proper treatment and management, some people with adenomatosis pulmonar may experience a good quality of life for many years.
Adenomatosis Pulmonar Complications
Complications of adenomatosis pulmonar can include:
* Respiratory failure
* Pneumonia or other infections
* Lung cancer
* Heart problems, such as arrhythmias and heart failure
* Malnutrition and weight loss
* Fatigue and weakness
Prevention
There is no known prevention for adenomatosis pulmonar. However, smoking cessation and avoiding exposure to environmental irritants can help reduce the risk of developing the condition. Early detection and treatment can also help improve outcomes.
Adenomatosis Pulmonar Procedure
There are several procedures that may be used to diagnose and treat adenomatosis pulmonar, including:
* Bronchoscopy: a flexible tube with a camera and light on the end is inserted through the nose or mouth to examine the airways and take biopsies.
* Thoracoscopy: a small incision is made in the chest, and a scope is inserted to examine the lungs and take biopsies.
* Lung biopsy: a sample of tissue is taken from the lung to examine under a microscope for cancer or other conditions.
* Pleurodesis: a procedure to seal the space between the lungs and chest wall to prevent fluid buildup.
Adenomatosis Pulmonar Treatment
Treatment for adenomatosis pulmonar depends on the underlying cause of the condition, but may include:
* Medications to manage symptoms such as coughing and difficulty breathing.
* Surgery to remove tumors or repair damaged tissue.
* Radiation therapy to kill cancer cells.
* Oxygen therapy to help improve oxygen levels in the blood.
Adenomatosis Pulmonar Prognosis
The prognosis for adenomatosis pulmonar is generally poor, as the condition can be difficult to diagnose and treat effectively. However, with early detection and appropriate treatment, some people may experience improved symptoms and quality of life.
Adenomatosis Pulmonar Complications
Complications of adenomatosis pulmonar may include:
* Respiratory failure
* Pneumonia or other infections
* Lung cancer
* Heart problems, such as arrhythmias and heart failure.
Adenomatosis Pulmonar Prevention
There is no known prevention for adenomatosis pulmonar, but early detection and treatment of underlying conditions can help to manage the condition and prevent complications.
Adenomatosis Pulmonar Living With
Living with adenomatosis pulmonar can be challenging, as the condition can cause a range of symptoms that can affect quality of life. However, with proper treatment and management, many people are able to lead active and fulfilling lives.
Adenomatosis Pulmonar Coping
Coping with adenomatosis pulmonar may involve a range of strategies, such as:
* Managing stress and anxiety through relaxation techniques and therapy.
* Staying active and engaging in regular exercise to improve overall health and well-being.
* Joining support groups to connect with others who are experiencing similar challenges.
Adenomatosis Pulmonar Support
Support for people living with adenomatosis pulmonar may include:
* Medications to manage symptoms such as coughing and difficulty breathing.
* Oxygen therapy to help improve oxygen levels in the blood.
* Lifestyle changes, such as quitting smoking and avoiding exposure to pollutants.
Adenomatosis Pulmonar Prognosis
The prognosis for adenomatosis pulmonar varies depending on the underlying cause of the condition and the severity of symptoms. In general, early detection and treatment can improve outcomes and reduce the risk of complications. However, in some cases, the condition can be severe and may lead to life-threatening complications such as respiratory failure.
Adenomatosis Pulmonar Treatment
Treatment for adenomatosis pulmonar typically involves a combination of medications and lifestyle changes. Medications may include:
* Bronchodilators to open up the airways and improve breathing.
* Corticosteroids to reduce inflammation in the lungs.
* Antibiotics to treat any underlying infections.
Lifestyle changes may include:
* Quitting smoking to reduce irritation to the lungs.
* Avoiding exposure to pollutants such as dust and chemicals.
* Getting regular exercise to improve lung function.
* Using a humidifier to add moisture to the air and relieve dryness in the lungs.
Adenomatosis Pulmonar Surgery
In severe cases of adenomatosis pulmonar, surgery may be necessary to remove damaged tissue from the lungs. The type of surgery used will depend on the severity of the condition and may include:
* Lobectomy, which involves removing a lobe of the lung that is affected by the condition.
* Pneumonectomy, which involves removing an entire lung.
Adenomatosis Pulmonar Prognosis
The prognosis for adenomatosis pulmonar varies depending on the underlying cause of the condition and the severity of symptoms. In general, early detection and treatment can improve outcomes and reduce the risk of complications. However, in some cases, the condition can be severe and may lead to life-threatening complications such as respiratory failure.
Adenomatosis Pulmonar Prevention
There is no known way to prevent adenomatosis pulmonar, but early detection and treatment of underlying conditions can help reduce the risk of developing the condition. Quitting smoking and avoiding exposure to pollutants such as dust and chemicals can also help reduce the risk of developing the condition.
* Definition: A blockage at the bladder neck that impedes urine flow.
* Causes: Tumors, stones, or inflammation can cause UBNO.
* Symptoms: Painful urination, frequency, and urgency.
* Diagnosis: Imaging tests like CT scans or ultrasounds may be used.
* Treatment: Surgery, chemotherapy, or radiation therapy may be necessary.
Note: This definition is an example and should not be considered a comprehensive or definitive definition of Urinary Bladder Neck Obstruction. The specifics of the condition may vary depending on the individual case.
Adenocarcinoma is a term used to describe a variety of different types of cancer that arise in glandular tissue, including:
1. Colorectal adenocarcinoma (cancer of the colon or rectum)
2. Breast adenocarcinoma (cancer of the breast)
3. Prostate adenocarcinoma (cancer of the prostate gland)
4. Pancreatic adenocarcinoma (cancer of the pancreas)
5. Lung adenocarcinoma (cancer of the lung)
6. Thyroid adenocarcinoma (cancer of the thyroid gland)
7. Skin adenocarcinoma (cancer of the skin)
The symptoms of adenocarcinoma depend on the location of the cancer and can include:
1. Blood in the stool or urine
2. Abdominal pain or discomfort
3. Changes in bowel habits
4. Unusual vaginal bleeding (in the case of endometrial adenocarcinoma)
5. A lump or thickening in the breast or elsewhere
6. Weight loss
7. Fatigue
8. Coughing up blood (in the case of lung adenocarcinoma)
The diagnosis of adenocarcinoma is typically made through a combination of imaging tests, such as CT scans, MRI scans, and PET scans, and a biopsy, which involves removing a sample of tissue from the affected area and examining it under a microscope for cancer cells.
Treatment options for adenocarcinoma depend on the location of the cancer and can include:
1. Surgery to remove the tumor
2. Chemotherapy, which involves using drugs to kill cancer cells
3. Radiation therapy, which involves using high-energy X-rays or other particles to kill cancer cells
4. Targeted therapy, which involves using drugs that target specific molecules on cancer cells to kill them
5. Immunotherapy, which involves using drugs that stimulate the immune system to fight cancer cells.
The prognosis for adenocarcinoma is generally good if the cancer is detected and treated early, but it can be more challenging to treat if the cancer has spread to other parts of the body.
There are many different types of polyps that can occur in various parts of the body, including:
1. Colon polyps: These are the most common type of polyp and typically occur in the colon or rectum. They are usually small and can be removed during a colonoscopy.
2. Thyroid polyps: These occur in the thyroid gland and are often benign. However, some can become cancerous if left untreated.
3. Nasal polyps: These occur in the nasal passages and are often associated with chronic sinusitis.
4. Ovarian polyps: These occur on the ovaries and are typically benign.
5. Uterine polyps: These occur in the uterus and are usually benign, but can occasionally become cancerous.
Polyps are often asymptomatic, meaning they do not cause any noticeable symptoms. However, some people may experience symptoms such as bleeding, abdominal pain, or difficulty swallowing if the polyp is large enough to interfere with normal bodily functions.
If you suspect you have a polyp, it is important to seek medical attention. Your healthcare provider will perform a physical examination and may order imaging tests such as an endoscopy or a CT scan to confirm the presence of the polyp. Treatment options for polyps depend on the type, size, and location of the polyp, as well as your overall health. Some polyps can be removed during an endoscopy or surgery, while others may require no treatment at all.
In summary, polyps are abnormal growths that can occur in various parts of the body. They are typically benign but can occasionally become cancerous if left untreated. If you suspect you have a polyp, it is important to seek medical attention for proper diagnosis and treatment.
Some common types of prostatic diseases include:
1. Benign Prostatic Hyperplasia (BPH): This is a non-cancerous enlargement of the prostate gland that can cause urinary frequency, weak stream, and other symptoms.
2. Acute Prostatitis: This is an inflammation of the prostate gland that can cause fever, chills, and painful urination.
3. Chronic Prostatitis: This is a long-term inflammation of the prostate gland that can cause ongoing symptoms such as painful urination and pelvic pain.
4. Prostate Cancer: This is a type of cancer that affects the prostate gland, and it is the second leading cause of cancer death in men.
5. Prostatodynia: This is a condition characterized by chronic pelvic pain and discomfort in the prostate area.
Prostatic diseases can be diagnosed through a variety of tests, including urinalysis, blood tests, and imaging studies such as ultrasound or MRI. Treatment options for prostatic diseases depend on the specific type of disease and its severity, and may include medications, surgery, or other interventions.
Prostatic diseases are common in men, especially as they age, and it is important for men to be aware of their risk factors and seek medical attention if they experience any symptoms. Regular check-ups with a healthcare provider can help to detect prostatic diseases early and prevent complications.
Symptoms of hyperaldosteronism may include high blood pressure, low potassium levels, muscle weakness, and heart arrhythmias. Treatment options vary depending on the underlying cause but may include medications to reduce aldosterone production, dietary modifications, and in some cases, surgery or radiation therapy.
It is important for individuals with hyperaldosteronism to receive regular monitoring and treatment from a healthcare provider to manage their condition effectively and prevent complications such as heart disease and stroke.
There are several types of prostatitis, including:
1. Acute bacterial prostatitis: This type is caused by a bacterial infection and can lead to fever, chills, and other symptoms.
2. Chronic bacterial prostatitis: This type is similar to acute bacterial prostatitis, but the infection lasts for more than 3 months.
3. Chronic abacterial prostatitis: This type is not caused by an infection and can be caused by irritation or inflammation of the prostate gland.
4. Asymptomatic inflammatory prostatitis: This type is characterized by inflammation of the prostate gland, but there are no symptoms.
Prostatitis can be diagnosed through a physical examination, urine tests, and imaging tests such as ultrasound or MRI. Treatment options for prostatitis depend on the type and severity of the condition, but may include antibiotics, alpha-blockers, and other medications to relieve symptoms.
In conclusion, prostatitis is a common condition that can cause a range of symptoms in men. It is important to seek medical attention if you experience any of the symptoms of prostatitis, as early diagnosis and treatment can help to alleviate discomfort and prevent complications.
Explanation: Neoplastic cell transformation is a complex process that involves multiple steps and can occur as a result of genetic mutations, environmental factors, or a combination of both. The process typically begins with a series of subtle changes in the DNA of individual cells, which can lead to the loss of normal cellular functions and the acquisition of abnormal growth and reproduction patterns.
Over time, these transformed cells can accumulate further mutations that allow them to survive and proliferate despite adverse conditions. As the transformed cells continue to divide and grow, they can eventually form a tumor, which is a mass of abnormal cells that can invade and damage surrounding tissues.
In some cases, cancer cells can also break away from the primary tumor and travel through the bloodstream or lymphatic system to other parts of the body, where they can establish new tumors. This process, known as metastasis, is a major cause of death in many types of cancer.
It's worth noting that not all transformed cells will become cancerous. Some forms of cellular transformation, such as those that occur during embryonic development or tissue regeneration, are normal and necessary for the proper functioning of the body. However, when these transformations occur in adult tissues, they can be a sign of cancer.
See also: Cancer, Tumor
Word count: 190
Intraductal carcinoma may or may not cause symptoms, and is usually detected by a mammogram. Treatment often involves surgery to remove the cancerous cells from the milk ducts. If left untreated, intraductal carcinoma may progress to more advanced breast cancer in some cases.
Intraductal carcinoma accounts for 20% of all breast cancers diagnosed each year in the United States, according to estimates from the American Cancer Society. The condition affects women of all ages, but is most common in postmenopausal women.
The adrenal cortex is a layer of tissue that surrounds the adrenal gland and produces steroid hormones, such as cortisol and aldosterone. Adrenal cortex diseases are conditions that affect the adrenal cortex and disrupt the normal production of these hormones.
Types of Adrenal Cortex Diseases:
1. Cushing's Syndrome: This is a condition caused by an excess of cortisol in the body, often due to a tumor on the adrenal gland. Symptoms include weight gain, high blood pressure, and diabetes.
2. Addison's Disease: This is a condition where the adrenal glands do not produce enough cortisol and aldosterone, often due to an autoimmune disorder or injury to the glands. Symptoms include fatigue, weight loss, and low blood pressure.
3. Adrenal Insuficiency: This is a condition where the adrenal glands do not produce enough cortisol and aldosterone, often due to a genetic disorder or injury to the glands. Symptoms include fatigue, weight loss, and low blood pressure.
4. Adrenal Hyperplasia: This is a condition where the adrenal glands produce too much cortisol, often due to a genetic disorder. Symptoms include high blood pressure, obesity, and diabetes.
5. Adrenocortical Carcinoma: This is a rare type of cancer that affects the adrenal cortex. Symptoms include weight loss, high blood pressure, and abdominal pain.
Treatment Options for Adrenal Cortex Diseases:
1. Medications: Depending on the specific condition, medications such as steroids, anti-inflammatory drugs, or hormone replacement therapy may be prescribed to manage symptoms.
2. Surgery: In some cases, surgery may be necessary to remove a tumor or affected tissue.
3. Lifestyle Changes: Patients with adrenal cortex diseases may need to make lifestyle changes such as reducing stress, losing weight, and increasing physical activity.
4. Hormone Replacement Therapy: This is often necessary in cases where the adrenal glands are not producing enough hormones.
5. Radiation Therapy: This is a treatment option for patients with adrenocortical carcinoma.
Prognosis for Adrenal Cortex Diseases:
The prognosis for adrenal cortex diseases varies depending on the specific condition and the severity of symptoms. In general, early diagnosis and treatment can improve outcomes. However, some conditions such as adrenocortical carcinoma can be difficult to treat and may have a poor prognosis.
Complications of Adrenal Cortex Diseases:
1. High Blood Pressure: Many adrenal cortex diseases can cause high blood pressure, which can lead to complications such as heart disease and stroke.
2. Hormonal Imbalances: Adrenal cortex diseases can disrupt the balance of hormones in the body, leading to a range of symptoms and complications.
3. Weight Gain or Loss: Some adrenal cortex diseases can cause weight gain or loss, which can lead to other health problems such as joint pain and decreased mobility.
4. Fatigue and Weakness: Adrenal cortex diseases can cause fatigue and weakness, making it difficult to perform daily activities.
5. Depression and Anxiety: Hormonal imbalances and other symptoms of adrenal cortex diseases can lead to depression and anxiety.
6. Increased Risk of Infections: Adrenal insufficiency can increase the risk of infections such as pneumonia and meningitis.
7. Decreased Immune Function: Hormonal imbalances can weaken the immune system, making it more difficult to fight off infections.
8. Adrenal Crisis: In rare cases, adrenal cortex diseases can lead to an adrenal crisis, which is a life-threatening condition that requires immediate medical attention.
Prevention of Adrenal Cortex Diseases:
While some adrenal cortex diseases may be genetic and unavoidable, there are steps you can take to prevent or manage the symptoms. Here are some ways to prevent adrenal cortex diseases:
1. Maintain a Balanced Diet: Eating a balanced diet that includes essential nutrients such as vitamins, minerals, and protein can help maintain healthy adrenal function.
2. Reduce Stress: High levels of stress can disrupt hormone production in the adrenal glands. Practicing stress-reducing techniques such as meditation, yoga, or deep breathing exercises can help manage stress.
3. Get Enough Sleep: Getting enough sleep is essential for maintaining healthy adrenal function. Aim for 7-8 hours of sleep per night.
4. Stay Hydrated: Drinking enough water throughout the day can help prevent dehydration, which can affect adrenal function.
5. Exercise Regularly: Regular exercise can help improve adrenal function and overall health. Aim for at least 30 minutes of moderate-intensity exercise per day.
6. Avoid Toxins: Exposure to toxins such as pesticides, heavy metals, and pollution can damage the adrenal glands. Avoiding these toxins by using non-toxic household cleaners, personal care products, and avoiding exposure to pollution can help prevent adrenal cortex diseases.
7. Manage Medications: Certain medications such as corticosteroids can disrupt the normal function of the adrenal glands. Monitoring and managing medications with your healthcare provider can help prevent adrenal cortex diseases.
8. Get Regular Check-Ups: Regular check-ups with your healthcare provider can help identify any potential issues before they become serious.
While some adrenal cortex diseases may be unavoidable, taking these steps can help prevent or manage symptoms and maintain overall health. If you experience any symptoms of adrenal cortex disease, it's essential to seek medical attention from a qualified healthcare provider for proper diagnosis and treatment.
1. Lymphedema: This is a condition in which the lymph vessels are unable to properly drain fluid from the body, leading to swelling in the affected limb.
2. Lymphangitis: This is an inflammation of the lymph vessels that can cause pain, redness, and swelling.
3. Lymphadenitis: This is an infection of the lymph nodes that can cause swelling, pain, and difficulty breathing.
4. Primary lymphedema: This is a rare genetic condition in which the lymph vessels are missing or do not develop properly.
5. Secondary lymphedema: This is a condition that develops as a result of another condition or injury, such as surgery, radiation therapy, or infection.
6. Lymphatic malformations: These are abnormalities in the development of the lymph vessels and nodes that can cause swelling, pain, and difficulty breathing.
7. Lymphocystis: This is a rare condition in which small cysts form in the lymph vessels and nodes.
8. Lymphangioleiomyomatosis (LAM): This is a rare condition that causes cysts to form in the lungs and can also affect the lymph vessels and nodes.
9. Lipedema: This is a condition in which there is an abnormal accumulation of fat in the legs, thighs, and buttocks, which can cause swelling and pain.
10. Pemphigus: This is a group of rare autoimmune disorders that affect the skin and mucous membranes, leading to blistering and scarring.
Treatment for lymphatic diseases depends on the specific condition and may include compression garments, exercises, and manual lymph drainage therapy. In some cases, medications such as antibiotics or anti-inflammatory drugs may be prescribed to help manage symptoms. Surgery may also be necessary in some cases to remove blockages or repair damaged vessels.
It is important to seek medical attention if you experience any persistent swelling or pain, as these can be signs of a lymphatic disease. Early diagnosis and treatment can help to manage symptoms and improve quality of life.
There are several types of angiomatosis, including:
1. Capillary angiomatosis: This type of angiomatosis is characterized by the growth of small blood vessels called capillaries. It can cause redness, itching, and swelling in the affected area.
2. Venous angiomatosis: This type of angiomatosis is characterized by the growth of veins, which can lead to varicose veins, bleeding, and swelling.
3. Lymphatic angiomatosis: This type of angiomatosis is characterized by the growth of lymph vessels, which can lead to swelling in the affected limb.
4. Arteriovenous malformation (AVM): This is a congenital condition where there is an abnormal connection between arteries and veins, leading to poor blood flow and oxygenation of tissues.
The symptoms of angiomatosis can vary depending on the location and size of the tumor. Some common symptoms include:
* Painless swelling or lump in the affected area
* Redness, itching, or bruising in the affected area
* Bleeding or discharge from the affected area
* Swelling in the limbs or other areas
* Fatigue or weakness
* Shortness of breath
The diagnosis of angiomatosis is based on a combination of physical examination, medical history, and imaging tests such as ultrasound, computed tomography (CT) scan, or magnetic resonance imaging (MRI). Treatment options for angiomatosis depend on the type and location of the tumor, and may include:
* Observation: Small, asymptomatic tumors may not require treatment and can be monitored with regular check-ups.
* Surgery: Larger tumors may require surgical removal to prevent complications such as bleeding or infection.
* Embolization: This is a minimally invasive procedure where a catheter is inserted through a blood vessel to deliver a substance that blocks the blood flow to the tumor, causing it to shrink.
* Radiation therapy: This may be used to treat tumors that are difficult to remove with surgery or embolization, or to relieve symptoms such as pain or bleeding.
* Medications: Depending on the type of angiomatosis, medications such as anti-inflammatory drugs, anticoagulants, or chemotherapy may be prescribed to manage symptoms and slow the growth of the tumor.
Overall, the prognosis for angiomatosis varies depending on the type and location of the tumor, as well as the promptness and effectiveness of treatment. With early diagnosis and appropriate treatment, many people with angiomatosis can lead normal lives and enjoy a good quality of life. However, in some cases, angiomatosis can be a serious condition that requires ongoing monitoring and treatment to prevent complications and manage symptoms.
The main difference between primary hyperparathyroidism (HPT) and secondary HPT is the underlying cause of the disorder. In primary HPT, the overactive parathyroid glands are due to a genetic mutation or an autoimmune response, while in secondary HPT, the overactivity is caused by another condition or medication that affects vitamin D levels.
The symptoms of SHPT are similar to those of primary HPT and may include:
* Bone pain or weakness
* Osteoporosis or osteopenia
* Kidney stones or other kidney problems
* High blood pressure
* Headaches
* Fatigue
* Nausea or vomiting
* Increased urination
SHPT can be diagnosed with a combination of physical examination, laboratory tests, and imaging studies such as ultrasound or CT scans. Treatment typically involves addressing the underlying cause of the condition and replacing vitamin D deficiency with supplements. In some cases, surgery may be necessary to remove part or all of the parathyroid glands.
While SHPT is rare, it is important for healthcare providers to be aware of this condition in patients who present with symptoms suggestive of HPT but have normal imaging studies and no family history of the condition. Early diagnosis and treatment can help prevent complications and improve quality of life for affected individuals.
In summary, secondary hyperparathyroidism is a rare endocrine disorder caused by a deficiency in vitamin D that leads to overactive parathyroid glands and an imbalance in calcium levels. It can cause a range of symptoms, including bone pain, osteoporosis, high blood pressure, and kidney problems. Treatment involves addressing the underlying cause of the condition and replacing vitamin D deficiency with supplements. Early diagnosis and treatment can help prevent complications and improve quality of life for affected individuals.
1. Difficulty starting to urinate (urinary hesitancy)
2. Weak or interrupted urine flow
3. Frequent urination
4. Urgency to urinate (feeling like you need to go right away)
5. Leaking of urine (incontinence)
6. Painful urination
7. Blood in the urine
8. Cloudy or strong-smelling urine
9. Recurrent urinary tract infections (UTIs)
10. Pelvic pain or discomfort
LUTS can be caused by a variety of factors, such as benign prostatic hyperplasia (BPH), bladder infection, kidney stones, interstitial cystitis, and neurological disorders. They can also be a symptom of certain medical conditions, such as diabetes, multiple sclerosis, and spinal cord injuries.
The evaluation and treatment of LUTS depend on the underlying cause and severity of the symptoms. A thorough medical history and physical examination are essential to diagnose the underlying cause of LUTS. Additional tests such as urine flow study, urinalysis, and imaging studies may be ordered to confirm the diagnosis. Treatment options for LUTS can include medications, lifestyle changes, and surgical interventions.
Some examples of pathologic constrictions include:
1. Stenosis: A narrowing or constriction of a blood vessel or other tubular structure, often caused by the buildup of plaque or scar tissue.
2. Asthma: A condition characterized by inflammation and constriction of the airways, which can make breathing difficult.
3. Esophageal stricture: A narrowing of the esophagus that can cause difficulty swallowing.
4. Gastric ring constriction: A narrowing of the stomach caused by a band of tissue that forms in the upper part of the stomach.
5. Anal fissure: A tear in the lining of the anus that can cause pain and difficulty passing stools.
Pathologic constrictions can be caused by a variety of factors, including inflammation, infection, injury, or genetic disorders. They can be diagnosed through imaging tests such as X-rays, CT scans, or endoscopies, and may require surgical treatment to relieve symptoms and improve function.
Some common types of adrenal gland neoplasms include:
1. Adrenocortical carcinoma: A rare and aggressive malignancy that arises in the outer layer of the adrenal cortex.
2. Adrenocortical adenoma: A benign tumor that arises in the outer layer of the adrenal cortex.
3. Pheochromocytoma: A rare tumor that arises in the inner part of the adrenal medulla and produces excessive amounts of hormones such as epinephrine and norepinephrine.
4. Paraganglioma: A rare tumor that arises in the sympathetic nervous system, often near the adrenal glands.
Symptoms of adrenal gland neoplasms can include:
* Weight gain or weight loss
* High blood pressure
* Fatigue
* Abdominal pain
* Headache
* Nausea and vomiting
* Palpitations
Diagnosis of adrenal gland neoplasms typically involves imaging tests such as computed tomography (CT) scans, magnetic resonance imaging (MRI), and positron emission tomography (PET) scans, as well as hormone level assessments. Treatment options vary depending on the type and size of the tumor, and may include surgery, chemotherapy, and hormone therapy.
Hyperplasia
Atypical hyperplasia
Uterine hyperplasia
Intimal hyperplasia
Thymus hyperplasia
Neuroendocrine hyperplasia
Microglandular hyperplasia
Neointimal hyperplasia
Lymphoid hyperplasia
Pneumocytic hyperplasia
Mesothelial hyperplasia
Sebaceous hyperplasia
Pulmonary hyperplasia
Follicular hyperplasia
Condylar hyperplasia
Endometrial hyperplasia
Nodular regenerative hyperplasia
Atypical adenomatous hyperplasia
Inflammatory papillary hyperplasia
Atypical ductal hyperplasia
Focal nodular hyperplasia
Salivary gland hyperplasia
Pseudoangiomatous stromal hyperplasia
Congenital adrenal hyperplasia
Alpha cell hyperplasia
Cutaneous lymphoid hyperplasia
Benign prostatic hyperplasia
Intravascular papillary endothelial hyperplasia
Pulmonary atypical adenomatous hyperplasia
Walleye epidermal hyperplasia virus
Congenital Adrenal Hyperplasia (CAH) | NICHD - Eunice Kennedy Shriver National Institute of Child Health and Human Development
Prostate Enlargement (Benign Prostatic Hyperplasia) - NIDDK
Non-classic congenital adrenal hyperplasia due to 21-hydroxylase deficiency - About the Disease - Genetic and Rare Diseases...
Congenital adrenal hyperplasia: MedlinePlus Medical Encyclopedia
Endothelial Hyperplasia - The Digitized Atlas of Mouse Liver Lesions
Nose, Epithelium - Hyperplasia - Nonneoplastic Lesion Atlas
Grant Abstract: Phytotherapy for Benign Prostatic Hyperplasia
Focal Nodular Hyperplasia (FNH) Imaging and Diagnosis: Practice Essentials, Computed Tomography, Magnetic Resonance Imaging
Congenital adrenal hyperplasia due to 11-beta-hydroxylase deficiency: MedlinePlus Genetics
Airway smooth muscle hypertrophy and hyperplasia in asthma
Azathioprine induced nodular regenerative hyperplasia in IBD patients - PubMed
Benign Prostate Hyperplasia - Prostate Imaging | UCLA Health
Conditions We Treat: Benign Prostatic Hyperplasia (BPH) | Johns Hopkins Brady Urological Institute
Women With Atypical Hyperplasia at Higher Risk of Breast Cancer
Endometrial hyperplasia CT - wikidoc
Congenital adrenal hyperplasia | DermNet
Insulinoma and Generalized Islet Cell Hyperplasia in a Patient with Diabetes Mellitus | SMJ
Gingival hyperplasia, hypertrichosis and a characteristic facies | Pediatric Oncall Journal
Feline Mammary Hyperplasia Resistant to Ovariohysterectomy: A Case Report - WSAVA2009 - VIN
Serum protein fingerprinting coupled with a pattern-matching algorithm distinguishes prostate cancer from benign prostate...
Cutaneous lymphoid hyperplasia developing on the site of a positive intradermal allergy test to cefotetan - Indian Journal of...
Esophagus - Hyperplasia - Nonneoplastic Lesion Atlas
Surgical laser on fibrous hyperplasia treatment
ARMC5-negative primary bilateral macronodular adrenal hyperplasia. | BMJ Case Rep;16(7)2023 Jul 06. | MEDLINE
Autoimmunity, Intestinal Lymphoid Hyperplasia, and Defects in Mucosal B-Cell Homeostasis in Patients With PTEN Hamartoma Tumor...
DailyMed - TERAZOSIN- terazosin hydrochloride capsule
Large pseudoangiomatous stromal hyperplasia complicated with gynecomastia and lobular differentiation in a male breast |...
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WHO HQ Library catalog
Congenital Adrenal Hyperplasia
Congenital32
- What causes congenital adrenal hyperplasia? (nih.gov)
- What are the symptoms of congenital adrenal hyperplasia? (nih.gov)
- How do health care providers diagnose congenital adrenal hyperplasia? (nih.gov)
- What are the treatments for congenital adrenal hyperplasia? (nih.gov)
- Links to websites of groups that study or provide information about congenital adrenal hyperplasia. (nih.gov)
- Congenital adrenal hyperplasia is the name given to a group of inherited disorders of the adrenal gland . (nih.gov)
- People with congenital adrenal hyperplasia lack an enzyme the adrenal glands need to make the hormones. (nih.gov)
- Congenital adrenal hyperplasia can affect both boys and girls. (nih.gov)
- About 1 in 10,000 to 18,000 children are born with congenital adrenal hyperplasia. (nih.gov)
- Symptoms will vary, depending on the type of congenital adrenal hyperplasia someone has, and their age when the disorder is diagnosed. (nih.gov)
- Children with milder forms may not have signs or symptoms of congenital adrenal hyperplasia and may not be diagnosed until as late as adolescence. (nih.gov)
- Steroids used to treat congenital adrenal hyperplasia do not usually cause side effects such as obesity or weak bones, because the doses replace the hormones that the child's body cannot make. (nih.gov)
- In some cases, congenital adrenal hyperplasia can affect fertility. (nih.gov)
- Parents with a family history of congenital adrenal hyperplasia (of any type) or a child who has the condition should consider genetic counseling. (nih.gov)
- Prenatal diagnosis is available for some forms of congenital adrenal hyperplasia. (nih.gov)
- A newborn screening test is available for the most common form of congenital adrenal hyperplasia. (nih.gov)
- Congenital adrenal hyperplasia (CAH) due to 11-beta-hydroxylase deficiency is one of a group of disorders (collectively called congenital adrenal hyperplasia) that affect the adrenal glands. (nih.gov)
- CAH due to 11-beta-hydroxylase deficiency accounts for 5 to 8 percent of all cases of congenital adrenal hyperplasia. (nih.gov)
- Congenital adrenal hyperplasia (CAH) is an autosomal recessive disease emerging from mutations of genes for enzymes that lead to the production of glucocorticoids, mineralocorticoids, and sex steroids by the adrenal glands. (nih.gov)
- This activity summarizes the evaluation and management of patients with congenital adrenal hyperplasia and highlights the role of the interprofessional team in managing this condition. (nih.gov)
- Describe the etiology of congenital adrenal hyperplasia. (nih.gov)
- Summarize the role of diagnostic imaging in the evaluation of congenital adrenal hyperplasia. (nih.gov)
- Outline the management of congenital adrenal hyperplasia. (nih.gov)
- Review the importance of collaboration and communication among interprofessional team members to enhance care coordination for patients with congenital adrenal hyperplasia. (nih.gov)
- Congenital adrenal hyperplasia most commonly results from mutations or deletions of CYP21A. (nih.gov)
- In the United States, congenital adrenal hyperplasia is more common in Native Americans and Yupik Eskimos. (nih.gov)
- Congenital adrenal hyperplasia is the most common adrenal disorder of infancy and childhood. (dermnetnz.org)
- Congenital adrenal hyperplasia results from excessive androgens (male hormones). (dermnetnz.org)
- How does congenital adrenal hyperplasia arise? (dermnetnz.org)
- Congenital adrenal hyperplasia may be contrasted with Cushings Syndrome , in which there is excess cortisol. (dermnetnz.org)
- The clinical signs of congenital adrenal hyperplasia depend on which enzyme is lacking and to what extent. (dermnetnz.org)
- Congenital generalized terminal hypertrichosis with gingival hyperplasia. (pediatriconcall.com)
Focal nodular hyp10
- There is uncertainty regarding the role, if any, of oral contraceptive steroids in the development of focal nodular hyperplasia of the liver. (nih.gov)
- A left hepatic lobectomy during the second trimester disclosed focal nodular hyperplasia. (nih.gov)
- Both contraceptive steroids and pregnancy, with high levels of endogenous sex steroids, favored tumor growth in this patient, suggesting that focal nodular hyperplasia can be steroid related. (nih.gov)
- Hepatic adenoma and focal nodular hyperplasia. (nih.gov)
- Fibrolamellar hepatocellular carcinoma arising in a background of focal nodular hyperplasia: a report of 2 cases. (nih.gov)
- Association of hepatocellular adenoma and focal nodular hyperplasia of the liver in a woman on oral contraceptives]. (nih.gov)
- Adenoma of the liver and focal nodular hyperplasia. (nih.gov)
- Focal nodular hyperplasia (FNH) is a non-neoplastic hepatic mass lesion and this is also the second most prevalent benign canine liver lesion. (petmoo.com)
- Focal nodular hyperplasia is defined as a localized, proliferative, hyperplastic overgrowth of the epithelial parenchymatous cell of the liver near a vascular anomaly, particularly an arterial irregularity. (petmoo.com)
- This is rare in focal nodular hyperplasia. (petmoo.com)
Lesions2
- Lesions of sebaceous hyperplasia are benign, with no known potential for malignant transformation, but they may be associated with nonmelanoma skin cancer in transplantation patients. (medscape.com)
- They found that as the extent of atypical hyperplasia in a biopsy increased, as measured by the number of separate atypia lesions or foci, so did the woman's risk of developing breast cancer. (medindia.net)
Atypical hyperplasia9
- Women with atypical hyperplasia (also known as atypia) are at higher risk of developing breast cancer in future than previously thought, according to a Mayo clinic study. (medindia.net)
- The findings could benefit more than 100,000 women diagnosed each year with atypical hyperplasia. (medindia.net)
- The research team recommends that women with atypical hyperplasia be recognized as having significantly increased lifetime risk of breast cancer and thus be candidates for intense screening with MRI. (medindia.net)
- Since atypia is also divided into simple and complex, the following types of endometrial hyperplasia is simple atypical hyperplasia. (netlify.app)
- Complex atypical hyperplasia endometrial cancers becomes approximately 25-30% of cases. (netlify.app)
- Mild form of complex atypical hyperplasia often amenable to progestin therapy. (netlify.app)
- According to one study, only 20% of cases of severe complex atypical hyperplasia treatable progestin, and often (in 25-45% of cases) after a hysterectomy in patients with this disorder discovered previously undiagnosed cancer of the endometrium. (netlify.app)
- For example, for women in the postmenopausal age, taking medications for hormone replacement therapy, hysterectomy may be the preferred method of treatment, as they have in the complex atypical hyperplasia increased risk of endometrial cancer. (netlify.app)
- If a woman regularly undergo a medical examination, and is closely monitoring the health, noting any change in his status, complex atypical hyperplasia and endometrial cancer will certainly be identified long before they become life-threatening. (netlify.app)
Endometrial hyperplasia3
- There are no CT findings associated with endometrial hyperplasia. (wikidoc.org)
- The results of recent research suggest that the diagnosis of the type of endometrial hyperplasia is necessary, first of all, to identify the following: whether simple or complex hyperplasia, as well as whether there are abnormal cells in the endometrium (in this case speak of atypia). (netlify.app)
- This species is also called adenomatous hyperplasia endometrial hyperplasia. (netlify.app)
Etiology1
- Nodular hyperplasia etiology is poorly understood. (petmoo.com)
Prostatic36
- What is benign prostatic hyperplasia? (nih.gov)
- Benign prostatic hyperplasia-also called BPH-is a condition in men in which the prostate gland is enlarged and not cancerous. (nih.gov)
- Benign prostatic hyperplasia is also called benign prostatic hypertrophy or benign prostatic obstruction. (nih.gov)
- Benign prostatic hyperplasia often occurs with the second growth phase. (nih.gov)
- The narrowing of the urethra and urinary retention-the inability to empty the bladder completely-cause many of the problems associated with benign prostatic hyperplasia. (nih.gov)
- Benign prostatic hyperplasia does not develop in men whose testicles were removed before puberty. (nih.gov)
- For this reason, some researchers believe factors related to aging and the testicles may cause benign prostatic hyperplasia. (nih.gov)
- Scientific studies have suggested that benign prostatic hyperplasia may occur because the higher proportion of estrogen within the prostate increases the activity of substances that promote prostate cell growth. (nih.gov)
- Scientists have noted that men who do not produce DHT do not develop benign prostatic hyperplasia. (nih.gov)
- How common is benign prostatic hyperplasia? (nih.gov)
- Benign prostatic hyperplasia is the most common prostate problem for men older than age 50. (nih.gov)
- In 2010, as many as 14 million men in the United States had lower urinary tract symptoms suggestive of benign prostatic hyperplasia. (nih.gov)
- 1 Although benign prostatic hyperplasia rarely causes symptoms before age 40, the occurrence and symptoms increase with age. (nih.gov)
- Benign prostatic hyperplasia affects about 50 percent of men between the ages of 51 and 60 and up to 90 percent of men older than 80. (nih.gov)
- Who is more likely to develop benign prostatic hyperplasia? (nih.gov)
- What are the symptoms of benign prostatic hyperplasia? (nih.gov)
- This Request for Applications (RFA), Treatment of Benign Prostatic Hyperplasia: Pilot Study, is related to the priority area of diabetes and chronic disabling diseases. (nih.gov)
- Experience in recruitment and follow-up of a large number of patients with urologic diseases in clinical trials as well as clinical experience with a large number of patients with benign prostatic hyperplasia, is especially useful for Clinical Centers. (nih.gov)
- RESEARCH OBJECTIVES Background Benign Prostatic Hyperplasia (BPH) is an abnormal, localized, enlargement of the prostate gland of adult men. (nih.gov)
- Abstract: DESCRIPTION (provided by applicant): The use of herbal extracts for treatment of benign prostatic hyperplasia (BPH) is growing rapidly in the United States. (nih.gov)
- Benign prostatic hyperplasia, or BPH, is a noncancerous enlargement of the prostate gland. (uclahealth.org)
- Benign prostatic hyperplasia (BPH) is a noncancerous enlargement of the prostate gland and the most common benign tumor found in men. (hopkinsmedicine.org)
- Benign Prostatic Hyperplasia Drugs Market analysis & forecast 2028 by Mia C. (apsense.com)
- Benign Prostatic Hyperplasia Drugs Market estimated to reach USD 20277.05 million by 2027 with expected CAGR of 8.4 % between 2019 to 2027. (apsense.com)
- Benign Prostatic Hyperplasia Drugs are the medical Drugs used for the treatment of Benign Prostatic Hyperplasia. (apsense.com)
- Benign Prostatic Hyperplasia Drugs historical, current and forecasted market size from the standpoint of value as well as volume. (apsense.com)
- Growth pattern, profitability and marketing assumptions are also important aspects of competitive analysis for Benign Prostatic Hyperplasia Drugs Market. (apsense.com)
- Study includes Top-down and bottom-up approaches in order to validate the global Benign Prostatic Hyperplasia Drugs size, regional analysis, product segments and end users /applications. (apsense.com)
- Market research study on Benign Prostatic Hyperplasia Drugs Market precisely focuses on key indicators of market growth. (apsense.com)
- Research study on Benign Prostatic Hyperplasia Drugs Market helps clients to make precise decision in order to expand their market share globally. (apsense.com)
- Benign prostatic hyperplasia (BPH) can lead to obstructive and bothersome lower urinary tract symptoms. (nih.gov)
- Benign prostatic hyperplasia is common in aging men, but rare in childhood. (yonsei.ac.kr)
- We report a case of juvenile prostatic hyperplasia in a 10-year-old boy, who visited our clinic for hematuria and voiding symptoms. (yonsei.ac.kr)
- Silodosin capsule, an alpha-1 adrenergic receptor antagonist, is indicated for the treatment of the signs and symptoms of benign prostatic hyperplasia (BPH). (nih.gov)
- Results of search for 'su:{Prostatic hyperplasia. (who.int)
- Benign Prostatic Hyperplasia (BPH) merupakan penyakit yang dikarakteristikan oleh pembesaran prostat. (indonesiare.co.id)
Nodular5
- Nodular regenerative hyperplasia rarely leads to liver transplantation: A 20-year cohort study in all Dutch liver transplant units. (nih.gov)
- Nodular regenerative hyperplasia of the liver associated with macroglobulinemia. (nih.gov)
- Nodular regenerative hyperplasia of the liver following bone marrow transplantation. (nih.gov)
- Micronodular transformation (nodular regenerative hyperplasia) of the liver: a report of 64 cases among 2,500 autopsies and a new classification of benign hepatocellular nodules. (nih.gov)
- The prognosis for Nodular hyperplasia is really good. (petmoo.com)
Benign prostate6
- Serum protein fingerprinting coupled with a pattern-matching algorithm distinguishes prostate cancer from benign prostate hyperplasia and healthy men. (nih.gov)
- Surface enhanced laser desorption/ionization mass spectrometry protein profiles of serum from 167 PCA patients, 77 patients with benign prostate hyperplasia, and 82 age-matched unaffected healthy men were used to train and develop a decision tree classification algorithm that used a nine-protein mass pattern that correctly classified 96% of the samples. (nih.gov)
- A sensitivity of 83%, a specificity of 97%, and a positive predictive value of 96% for the study population and 91% for the general population were obtained when comparing the PCA versus noncancer (benign prostate hyperplasia/healthy men) groups. (nih.gov)
- Benign Prostate Hyperplasia (BPH) is a condition more commonly known as enlarged prostate gland. (riaendovascular.com)
- What imaging/tests are done to diagnose benign prostate hyperplasia? (riaendovascular.com)
- What intervention/treatment can be performed for benign prostate hyperplasia? (riaendovascular.com)
Hypertrophy6
- Hyperplasia of the sebaceous cells is often accompanied by hypertrophy. (nih.gov)
- Hyperplasia is different from hypertrophy in that the adaptive cell change in hypertrophy is an increase in the size of cells, whereas hyperplasia involves an increase in the number of cells. (dbpedia.org)
- Increased thickness of the airway smooth muscle (ASM) layer in asthma may result from hyperplasia or hypertrophy of muscle cells or increased extracellular matrix (ECM). (nih.gov)
- To relate ASM hypertrophy, ASM hyperplasia, and deposition of ECM to the severity and duration of asthma. (nih.gov)
- Hypertrophy of ASM cells occurs in the large airways in both nonfatal and fatal cases of asthma, but hyperplasia of ASM cells is present in the large and small airways in fatal asthma cases only. (nih.gov)
- Duration of asthma has little or no effect on ASM hypertrophy or hyperplasia or f(ECM). (nih.gov)
Pathology2
- When follicular epithelium is also hyperplastic, this should be reflected in the severity grade assigned to epithelial hyperplasia and described in the pathology narrative. (nih.gov)
- Primary bilateral macronodular adrenal hyperplasia was confirmed by pathology after bilateral adrenalectomy . (bvsalud.org)
Patients2
- Sebaceous hyperplasia and skin cancer in patients undergoing renal transplant. (medscape.com)
- Premature familial sebaceous hyperplasia: successful response to oral isotretinoin in three patients. (medscape.com)
Deficiency1
- This mutation produces a 21-hydroxylase deficiency in 90%-95% of adrenal hyperplasia cases. (nih.gov)
Sebaceous glands3
- Sebaceous hyperplasia is a common, benign condition of sebaceous glands in adults of middle age or older. (medscape.com)
- In more severe cases, especially when accompanied by inflammation, hyperplasia of follicular epithelium, follicular units, and sebaceous glands often occurs. (nih.gov)
- Sebaceous gland hyperplasia is characterized by large sebaceous glands with increased numbers of cells forming numerous lobules around central ducts (Figure 3 and Figure 4). (nih.gov)
Hyperplastic1
- Sebaceous hyperplasia in organ transplant recipients: shared aspects of hyperplastic and dysplastic processes? (medscape.com)
Atypia3
- Atypia may be present in the simple and complex hyperplasia, so the total is divided into four main types of violations. (netlify.app)
- With a simple hyperplasia without atypia observed cell proliferation, but the basic structure of the endometrium remained relatively unchanged. (netlify.app)
- without atypia individual endometrial cancer may be increased, but, as with simple hyperplasia without atypia, the internal structure of cells remains normal. (netlify.app)
Treatment1
- Successful treatment of cyclosporine-induced sebaceous hyperplasia with oral isotretinoin in two renal transplant recipients. (medscape.com)
Lesion2
- Epithelial hyperplasia is the most common spontaneous, non-neoplastic lesion of the skin observed in B6C3F1 mice in NTP studies. (nih.gov)
- Pseudoangiomatous stromal hyperplasia (PASH) is a benign lesion often observed in parts of the mammary stroma in a variety of breast diseases. (springeropen.com)
Familial1
- Dupre A, Bonafe JL, Lamon P. Functional familial sebaceous hyperplasia of the face and premature sebaceous gland hyperplasia: a new and unique entity. (medscape.com)
Entity3
- 1-4) Canun from Mexico described an 11 years old girl who had gingival hyperplasia, hypertrichosis with a coarse and typical facies and proposed that this was a separate entity. (pediatriconcall.com)
- Canun et al proposed that this association of hypertrichosis, gingival hyperplasia and a characteristic facies is a separate entity. (pediatriconcall.com)
- Canun S, Guevara-Sangines EG, Elvira-Morales A, Sierra-Romero Mdel C, Rodriguez-Asbun H. Hypertrichosis terminalis, gingival hyperplasia, and a characteristic face: a new distinct entity. (pediatriconcall.com)
Occurs1
- Ultraviolet radiation is considered only a cofactor, because sebaceous hyperplasia occasionally occurs on areas of the body where sunlight is not a relevant issue, including the buccal mucosa, areolae, and vulva. (medscape.com)
Common3
- Cyclosporine-induced sebaceous hyperplasia in a hematopoetic stem cell transplant patient: delayed onset of a common adverse event. (medscape.com)
- Hyperplasia of the epithelium of the epidermis and adnexa is also a common response to dermal application of chemicals. (nih.gov)
- Hyperplasia is a common preneoplastic response to stimulus. (dbpedia.org)
Cases1
- Mandal RK, Das A, Chakrabarti I, Agarwal P. Nevoid sebaceous hyperplasia mistaken as nevus sebaceous: Report of four cases. (medscape.com)
Uncommon1
- Primary bilateral macronodular adrenal hyperplasia is an uncommon cause of endogenous Cushing syndrome . (bvsalud.org)
Affects1
- Feline mammary gland hyperplasia is a disease that consists of a localized or diffuse enlargement of one or more mammary glands and it affects young females both cycling or pregnant queens. (vin.com)
Regenerative1
- When present as a regenerative response, secondary to an adjacent ulcer, hyperplasia should not be diagnosed but should be described in the narrative. (nih.gov)
Severity2
Oral1
- Eruptive sebaceous hyperplasia as a side effect of oral tacrolimus in a renal transplant recipient. (medscape.com)
Clinical1
- Objective: to demonstrate through a clinical case report the excision of an inflammatory fibrous hyperplasia (IFH) with the use of surgical diode laser. (bvsalud.org)
Facial1
- Facial papular sebaceous hyperplasia is thought to be caused by a decrease in the circulating levels of androgen associated with aging. (medscape.com)
Malignant1
- Over the period of a 2-year study, sebaceous hyperplasia has the potential to progress to benign and malignant sebaceous cell neoplasms. (nih.gov)
Disease1
- Women who want to preserve the uterus and possibly the ability to have children in the future, you should know that even in this type of hyperplasia are several sub-types - from mild to severe forms of the disease. (netlify.app)
Chest2
- Sebaceous hyperplasia of the chest. (medscape.com)
- Sato T, Tanaka M. Linear sebaceous hyperplasia on the chest. (medscape.com)
Mental1
- 8) Gingival hyperplasia, hypertrichosis, with Mental retardation and epilepsy was reported by Anavi (9). (pediatriconcall.com)