Impaired ambulation not attributed to sensory impairment or motor weakness. FRONTAL LOBE disorders; BASAL GANGLIA DISEASES (e.g., PARKINSONIAN DISORDERS); DEMENTIA, MULTI-INFARCT; ALZHEIMER DISEASE; and other conditions may be associated with gait apraxia.
A group of cognitive disorders characterized by the inability to perform previously learned skills that cannot be attributed to deficits of motor or sensory function. The two major subtypes of this condition are ideomotor (see APRAXIA, IDEOMOTOR) and ideational apraxia, which refers to loss of the ability to mentally formulate the processes involved with performing an action. For example, dressing apraxia may result from an inability to mentally formulate the act of placing clothes on the body. Apraxias are generally associated with lesions of the dominant PARIETAL LOBE and supramarginal gyrus. (From Adams et al., Principles of Neurology, 6th ed, pp56-7)
A form of apraxia characterized by an acquired inability to carry out a complex motor activity despite the ability to mentally formulate the action. This condition has been attributed to a disruption of connections between the dominant parietal cortex and supplementary and premotor cortical regions in both hemispheres. (From Adams et al., Principles of Neurology, 6th ed, p57)
NECROSIS occurring in the ANTERIOR CEREBRAL ARTERY system, including branches such as Heubner's artery. These arteries supply blood to the medial and superior parts of the CEREBRAL HEMISPHERE, Infarction in the anterior cerebral artery usually results in sensory and motor impairment in the lower body.
Manner or style of walking.
Gait abnormalities that are a manifestation of nervous system dysfunction. These conditions may be caused by a wide variety of disorders which affect motor control, sensory feedback, and muscle strength including: CENTRAL NERVOUS SYSTEM DISEASES; PERIPHERAL NERVOUS SYSTEM DISEASES; NEUROMUSCULAR DISEASES; or MUSCULAR DISEASES.
Impairment of the ability to coordinate the movements required for normal ambulation (WALKING) which may result from impairments of motor function or sensory feedback. This condition may be associated with BRAIN DISEASES (including CEREBELLAR DISEASES and BASAL GANGLIA DISEASES); SPINAL CORD DISEASES; or PERIPHERAL NERVOUS SYSTEM DISEASES.
The use of music as an adjunctive therapy in the treatment of neurological, mental, or behavioral disorders.
Sound that expresses emotion through rhythm, melody, and harmony.
The scientific disciplines concerned with the embryology, anatomy, physiology, biochemistry, pharmacology, etc., of the nervous system.
Facilities which provide programs for rehabilitating the mentally or physically disabled individuals.
Experiential, attitudinal, emotional, or behavioral phenomena occurring during the course of treatment. They apply to the patient or therapist (i.e., nurse, doctor, etc.) individually or to their interaction. (American Psychological Association: Thesaurus of Psychological Index Terms, 1994)
Medical practice or discipline that is based on the knowledge, cultures, and beliefs of the people of KOREA.
Traumatic injuries to the brain, cranial nerves, spinal cord, autonomic nervous system, or neuromuscular system, including iatrogenic injuries induced by surgical procedures.

Gait apraxia after bilateral supplementary motor area lesion. (1/14)

OBJECTIVES: The study aimed at addressing the issue of the precise nature of gait apraxia and the cerebral dysfunction responsible for it. METHODS: The case of a patient, affected by a bilateral infarction limited to a portion of the anterior cerebral artery territory is reported. The patient's ability to walk was formally assessed by means of a new standardised test. RESULTS: Due to an anomaly within the anterior cerebral artery system, the patient's lesion was centred on the supplementary motor regions of both hemispheres. He presented with clear signs of gait apraxia that could not be accounted for by paresis or other neurological deficits. No signs of any other form of apraxia were detected. CONCLUSIONS: The clinical profile of the patient and the analysis of 49 cases from previous literature suggest that gait apraxia should be considered a clinical entity in its own right and lesions to the supplementary motor areas are responsible for it.  (+info)

Paraneoplastic chorea: case study with autopsy confirmation. (2/14)

A 67-year-old man presented with a 7-month history of insidiously progressive chorea, ataxia, and vertigo. Neurologic examination revealed deficits referable to the basal nuclei, cerebellar vermis, and vestibular nuclei. Small-cell lung cancer was diagnosed by fine-needle biopsy of a parahilar mass. After chemotherapy, the patient's chorea worsened. Anti-Hu antibodies were present in serum and cerebrospinal fluid. Microscopic examination of the brain at autopsy revealed diffuse perivascular lymphocytic infiltrates, microglial activation, and neuronophagia throughout the neuraxis, including the brainstem, cerebellum, lenticular nuclei, striatum, and cerebral cortex. Prominent loss of Purkinje cells was seen in the cerebellar vermis and hemispheres to a lesser degree. Chorea is extremely rare as a paraneoplastic manifestation of cancer. The florid presentation and the positive findings contrasted with an unremarkable MRI of the brain. This case illustrates the preeminence of symptoms and signs over negative MRI findings in paraneoplastic encephalitis.  (+info)

A new classification of higher level gait disorders in patients with cerebral multi-infarct states. (3/14)

BACKGROUND: cerebral multi-infarct states may lead to gait disorders in the absence of cognitive impairment. Where these gait disorders occur in the absence of neurological signs they have been termed gait apraxia or more recently higher-level gait disorders. In this paper we hypothesise three main types based on presumptive sites of anatomical damage: (a) Ignition Apraxia, where damage is predominantly in the supplementary motor area and its connections, with good responses to external clues; (b) Equilibrium Apraxia, where damage is predominantly in the pre-motor area in its connections, with poor responses to external cues and (c) Mixed Gait Apraxia. SUBJECTS: the clinical features and measured gait parameters of 13 patients with cerebral multi-infarct states and higher-level gait disorder are described (7 with Ignition Apraxia and 6 with Equilibrium Apraxia) along with those of 6 healthy elderly control subjects. METHODS: baseline gait characteristics were assessed on a walkway, which measured the following: step lengths, width of base and velocity. RESULTS: measured baseline gait parameters support the above hypothesis. CONCLUSIONS: it is suggested, though not proven, that patients with Ignition Apraxia could have problems with internal cueing due to lesions in the supplementary motor area or its connections whereas those with Equilibrium Apraxia could have dysfunction predominantly in the pre-motor area and its connections.  (+info)

Walking difficulties in patients with Alzheimer's disease might originate from gait apraxia. (4/14)

OBJECTIVES: To investigate whether gait apraxia is a possible cause for some of the walking abnormalities shown by patients with Alzheimer's disease. METHODS: 60 patients with Alzheimer's disease, selected as being free from overt extrapyramidal impairment or other potential causes of walking deficits, were assessed with a new test evaluating aspects of walking and related movements. Norms for this test were collected from a sample of 182 healthy volunteers. RESULTS: 40% of the Alzheimer group performed below the cut off score on this test, and half performed poorly. Performance of the Alzheimer group in the walking skills test correlated highly with scores in a test assessing limb apraxia and with dementia severity. CONCLUSIONS: Gait apraxia may be the cause of walking disorders found in a subgroup of patients with Alzheimer's disease. Its detection is made easier by the use of a standardised test, but still relies heavily on the exclusion of other causes of walking deficits. It is a recognisable and independent form of apraxia.  (+info)

Apraxia in movement disorders. (5/14)

The definition of apraxia specifies that the disturbance of performed skilled movements cannot be explained by the more elemental motor disorders typical of patients with movement disorders. Generally this does not present a significant diagnostic problem when dealing with 'higher-level' praxic disturbances (e.g. ideational apraxia), but it can be a major confound in establishing the presence of limb-kinetic apraxia. Most motor disturbances characteristic of extrapyramidal disorders, particularly bradykinesia and dystonia, will compromise the ability to establish the presence of loss of dexterity and deftness that constitutes this subtype. The term 'apraxia' has also been applied to other motor disturbances, such as 'gait apraxia' and 'apraxia of eyelid opening', that perhaps are misnomers, demonstrating the lack of a coherent nomenclature in this field. Apraxia is a hallmark of corticobasal degeneration (CBD) and historically this has received the most attention among the movement disorders. Corticobasal degeneration is characterized by various forms of apraxia affecting limb function, particularly ideomotor apraxia and limb-kinetic apraxia, although buccofacial and oculomotor apraxia can be present as well. The syndrome of parkinsonism and prominent apraxia, designated the 'corticobasal syndrome' (CBS), may be caused by a variety of other central nervous system pathologies including progressive supranuclear palsy (PSP), Alzheimer's disease, dementia with Lewy bodies and frontotemporal dementias. Distinct from the CBS, PSP and Parkinson's disease can demonstrate varying degrees of apraxia on selected tests, especially in those patients with more severe cognitive dysfunction. Diseases that cause the combination of apraxia and a primary movement disorder most often involve a variety of cerebral cortical sites as well as basal ganglia structures. Clinical-pathological correlates and functional imaging studies are compromised by both this diffuse involvement and the confusion experienced in the clinical evaluation of apraxia in the face of the additional elemental movement disorders. Finally, although apraxia results in clear disability in patients with the CBS, it is not clear how milder ideomotor apraxia found on specific testing contributes to patients' overall day-to-day motor disability.  (+info)

Lengthening and transfer of hamstrings for a flexion deformity of the knee in children with bilateral cerebral palsy: technique and preliminary results. (6/14)

Between July 2000 and April 2004, 19 patients with bilateral spastic cerebral palsy who required an assistive device to walk had combined lengthening-transfer of the medial hamstrings as part of multilevel surgery. A standardised physical examination, measurement of the Functional Mobility Scale score and video or instrumented gait analysis were performed pre- and post-operatively. Static parameters (popliteal angle, flexion deformity of the knee) and sagittal knee kinematic parameters (knee flexion at initial contact, minimum knee flexion during stance, mean knee flexion during stance) were recorded. The mean length of follow-up was 25 months (14 to 45). Statistically significant improvements in static and dynamic outcome parameters were found, corresponding to improvements in gait and functional mobility as determined by the Functional Mobility Scale. Mild hyperextension of the knee during gait developed in two patients and was controlled by adjustment of their ankle-foot orthosis. Residual flexion deformity > 10 degrees occurred in both knees of one patient and was treated by anterior distal femoral physeal stapling. Two children also showed an improvement of one level in the Gross Motor Function Classification System.  (+info)

Association between the metabolic syndrome and its components and gait speed among U.S. adults aged 50 years and older: a cross-sectional analysis. (7/14)

BACKGROUND: To examine the relationship between the metabolic syndrome and its components and gait speed among older U.S. men and women. Whether these associations are independent of physical activity was also explored. METHODS: Eight hundred and thirty-five men and 850 women aged > or =50 years from the continuous National Health and Nutrition Examination Survey 1999-2002 were examined. We used the definition of the metabolic syndrome developed by the U.S. National Cholesterol Education Program Adult Treatment Panel III. Gait speed was measured with a 6.10-meter timed walk examination. RESULTS: The prevalence of the metabolic syndrome was 40.2% in men and 45.6% in women (P = .127). The prevalence of gait speed impairment was 29.3% in men and 12.5% in women (P < .001). No association was found between the metabolic syndrome and gait speed impairment. After including the individual components of the metabolic syndrome in a logistic model adjusted for age and leisure-time physical activity, abdominal obesity, low HDL cholesterol, and high fasting glucose were significantly associated with gait speed impairment among women (adjusted odds ratio [AOR] = 0.48, 95% confidence interval [CI] = 0.26 to 0.89; AOR = 2.26, 95% CI = 1.08 to 4.75; and AOR = 2.05, 95% CI = 1.12 to 3.74, respectively). Further adjustment for race/ethnicity, education, smoking status, alcohol consumption, arthritis status, and use of an assistive device attenuated these associations; among women, abdominal obesity and low HDL cholesterol remained significantly associated with gait speed impairment (AOR = 0.37, 95% CI = 0.18 to 0.76 and AOR = 2.45, 95% CI = 1.07 to 5.63, respectively) while the association between hyperglycemia and impaired gait speed attenuated to nonsignificance. CONCLUSION: Among women, gait speed impairment is associated with low HDL cholesterol and inversely with abdominal obesity. These associations may be sex-dependent and warrant further research.  (+info)

Backward walking in Parkinson's disease. (8/14)

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Gait apraxia is a neurological disorder that affects an individual's ability to perform coordinated and complex movements required for walking, despite having the physical capability to do so. It is not caused by weakness or sensory loss, but rather by damage to the brain areas responsible for motor planning and execution, particularly in the frontal lobes.

Gait apraxia is characterized by a wide-based, hesitant, and unsteady gait pattern. Individuals with this condition may have difficulty initiating walking, changing direction, or adjusting their stride length and speed. They may also exhibit symptoms such as freezing of gait, where they are unable to move their feet forward despite intending to walk.

This disorder is often associated with various neurological conditions, including cerebrovascular accidents (strokes), degenerative diseases such as Parkinson's disease and multiple sclerosis, traumatic brain injuries, and infections of the central nervous system. Treatment typically involves physical therapy, gait training, and the use of assistive devices to improve mobility and safety.

Apraxia is a motor disorder characterized by the inability to perform learned, purposeful movements despite having the physical ability and mental understanding to do so. It is not caused by weakness, paralysis, or sensory loss, and it is not due to poor comprehension or motivation.

There are several types of apraxias, including:

1. Limb-Kinematic Apraxia: This type affects the ability to make precise movements with the limbs, such as using tools or performing complex gestures.
2. Ideomotor Apraxia: In this form, individuals have difficulty executing learned motor actions in response to verbal commands or visual cues, but they can still perform the same action when given the actual object to use.
3. Ideational Apraxia: This type affects the ability to sequence and coordinate multiple steps of a complex action, such as dressing oneself or making coffee.
4. Oral Apraxia: Also known as verbal apraxia, this form affects the ability to plan and execute speech movements, leading to difficulties with articulation and speech production.
5. Constructional Apraxia: This type impairs the ability to draw, copy, or construct geometric forms and shapes, often due to visuospatial processing issues.

Apraxias can result from various neurological conditions, such as stroke, brain injury, dementia, or neurodegenerative diseases like Parkinson's disease and Alzheimer's disease. Treatment typically involves rehabilitation and therapy focused on retraining the affected movements and compensating for any residual deficits.

Ideomotor apraxia is a neurological disorder that affects the ability to perform learned, purposeful movements in the absence of muscle weakness or paralysis. It results from damage to specific areas of the brain that are responsible for motor planning and execution.

In ideomotor apraxia, the person has difficulty translating an intention or idea into the appropriate movement. For example, if asked to pantomime using a toothbrush, they may not be able to recall and execute the correct sequence of movements required for this task, even though they understand what is being asked of them and have no problem moving their arm or hand.

This disorder can manifest as awkward, poorly coordinated, or incomplete movements, often with inconsistent errors. Ideomotor apraxia is typically seen following lesions to the left hemisphere of the brain, particularly in regions associated with language and motor function, such as Broca's area and the parietal lobe. Treatment usually involves occupational therapy and strategies to help compensate for the impaired motor skills.

Anterior cerebral artery infarction refers to the death of brain tissue (also known as an infarct) in the territory supplied by the anterior cerebral artery (ACA) due to insufficient blood flow. The ACA supplies oxygenated blood to the frontal lobes of the brain, which are responsible for higher cognitive functions such as reasoning, problem-solving, and decision-making, as well as motor control of the lower extremities.

An infarction in this territory can result from various causes, including atherosclerosis, embolism, thrombosis, or vasospasm. Symptoms of an ACA infarction may include weakness or paralysis on one side of the body (usually the lower extremities), difficulty with coordination and balance, urinary incontinence, changes in personality or behavior, and impaired cognitive function. The severity of symptoms depends on the extent and location of the infarct. Immediate medical attention is necessary to prevent further damage and improve the chances of recovery.

Gait is a medical term used to describe the pattern of movement of the limbs during walking or running. It includes the manner or style of walking, including factors such as rhythm, speed, and step length. A person's gait can provide important clues about their physical health and neurological function, and abnormalities in gait may indicate the presence of underlying medical conditions, such as neuromuscular disorders, orthopedic problems, or injuries.

A typical human gait cycle involves two main phases: the stance phase, during which the foot is in contact with the ground, and the swing phase, during which the foot is lifted and moved forward in preparation for the next step. The gait cycle can be further broken down into several sub-phases, including heel strike, foot flat, midstance, heel off, and toe off.

Gait analysis is a specialized field of study that involves observing and measuring a person's gait pattern using various techniques, such as video recordings, force plates, and motion capture systems. This information can be used to diagnose and treat gait abnormalities, improve mobility and function, and prevent injuries.

A gait disorder is a disturbance in the ability to walk that can't be attributed to physical disabilities such as weakness or paralysis. Neurologic gait disorders are those specifically caused by underlying neurological conditions. These disorders can result from damage to the brain, spinal cord, or peripheral nerves that disrupts communication between the muscles and the brain.

Neurologic gait disorders can present in various ways, including:

1. **Spastic Gait:** This is a stiff, foot-dragging walk caused by increased muscle tone (hypertonia) and stiffness (spasticity). It's often seen in conditions like cerebral palsy or multiple sclerosis.

2. **Ataxic Gait:** This is a broad-based, unsteady, and irregular walk caused by damage to the cerebellum, which affects balance and coordination. Conditions such as cerebellar atrophy or stroke can cause this type of gait disorder.

3. **Parkinsonian Gait:** This is a shuffling walk with small steps, flexed knees, and difficulty turning. It's often seen in Parkinson's disease.

4. **Neuropathic Gait:** This is a high-stepping walk caused by foot drop (difficulty lifting the front part of the foot), which results from damage to the peripheral nerves. Conditions such as diabetic neuropathy or Guillain-Barre syndrome can cause this type of gait disorder.

5. **Choreic Gait:** This is an irregular, dance-like walk caused by involuntary movements (chorea) seen in conditions like Huntington's disease.

6. **Mixed Gait:** Sometimes, a person may exhibit elements of more than one type of gait disorder.

The specific type of gait disorder can provide important clues about the underlying neurological condition and help guide diagnosis and treatment.

Gait ataxia is a type of ataxia, which refers to a lack of coordination or stability, specifically involving walking or gait. It is characterized by an unsteady, uncoordinated, and typically wide-based gait pattern. This occurs due to dysfunction in the cerebellum or its connecting pathways, responsible for maintaining balance and coordinating muscle movements.

In gait ataxia, individuals often have difficulty with controlling the rhythm and pace of their steps, tend to veer or stagger off course, and may display a reeling or stumbling motion while walking. They might also have trouble performing rapid alternating movements like quickly tapping their foot or heel. These symptoms are usually worse when the person is tired or attempting to walk in the dark.

Gait ataxia can be caused by various underlying conditions, including degenerative neurological disorders (e.g., cerebellar atrophy, multiple sclerosis), stroke, brain injury, infection (e.g., alcoholism, HIV), or exposure to certain toxins. Proper diagnosis and identification of the underlying cause are essential for effective treatment and management of gait ataxia.

Music therapy is a clinical and evidence-based use of music interventions to accomplish individualized goals within a therapeutic relationship by a credentialed professional who has completed an approved music therapy program.

The American Music Therapy Association (AMTA) defines music therapy as the "use of musical experiences, either created or selected, by a trained music therapist to help clients improve or restore physical, psychological, and social functions." The aim is to facilitate positive changes in emotional well-being, cognitive functioning, sensory-motor skills, communication, and quality of life.

Music therapists use various techniques such as singing, playing instruments, listening to music, and discussing lyrics to engage clients in the therapeutic process. These interventions can be tailored to meet the unique needs and preferences of each individual, making it a versatile form of therapy that can benefit people of all ages and abilities.

I'm sorry for any confusion, but "music" is not a term that has a medical definition. Music is a form of art that uses sound organized in time. It may include elements such as melody, harmony, rhythm, and dynamics. While music can have various psychological and physiological effects on individuals, it is not considered a medical term with a specific diagnosis or treatment application. If you have any questions related to medicine or health, I'd be happy to try to help answer those for you!

Neurosciences is a multidisciplinary field of study that focuses on the structure, function, development, and disorders of the nervous system, which includes the brain, spinal cord, and peripheral nerves. It incorporates various scientific disciplines such as biology, chemistry, physics, mathematics, engineering, and computer science to understand the complexities of the nervous system at different levels, from molecular and cellular mechanisms to systems and behavior.

The field encompasses both basic research and clinical applications, with the aim of advancing our knowledge of the nervous system and developing effective treatments for neurological and psychiatric disorders. Specialties within neurosciences include neuroanatomy, neurophysiology, neurochemistry, neuropharmacology, neurobiology, neuroimmunology, behavioral neuroscience, cognitive neuroscience, clinical neuroscience, and computational neuroscience, among others.

Rehabilitation centers are healthcare facilities that provide specialized therapeutic programs and services to individuals who are recovering from physical injuries, disabilities, or addictions. The main goal of rehabilitation centers is to help patients regain their independence, improve their functional abilities, and enhance their quality of life. These centers offer a multidisciplinary approach to care, often involving medical professionals such as physicians, nurses, therapists, psychologists, and social workers. Rehabilitation programs may include various forms of therapy, such as physical therapy, occupational therapy, speech-language pathology, recreational therapy, and psychological counseling. Additionally, rehabilitation centers may also provide education, support groups, and case management services to assist patients in their recovery process and help them reintegrate into their communities.

Psychotherapeutic processes refer to the methods and techniques used in psychotherapy to help individuals understand and overcome their emotional, cognitive, or behavioral issues. These processes involve various elements such as:

1. Establishing a therapeutic relationship: Building trust and rapport between the therapist and the client is crucial for successful therapy. This relationship provides a safe and supportive environment where the client can explore their thoughts and feelings.

2. Assessment and diagnosis: The psychotherapist evaluates the client's mental health status, identifies any underlying issues or disorders, and develops an individualized treatment plan based on this information.

3. Psychological interventions: These are specific techniques used to address the client's concerns, such as cognitive-behavioral therapy (CBT), psychodynamic therapy, or humanistic therapy. Each approach has its own unique focus, goals, and methods for helping clients change negative patterns of thinking, feeling, and behaving.

4. Collaborative goal setting: Both the therapist and client work together to establish clear, realistic goals for treatment that align with the client's needs and values.

5. Self-exploration and self-understanding: Through various therapeutic techniques, clients gain insights into their thoughts, emotions, and behaviors, allowing them to better understand themselves and their motivations.

6. Emotional regulation and coping skills development: Clients learn strategies to manage their emotions more effectively and cope with stressors in healthier ways.

7. Problem-solving and decision-making: Therapists help clients develop critical thinking skills and problem-solving abilities, enabling them to make better choices and navigate challenges in their lives.

8. Personal growth and change: As clients progress through therapy, they may experience positive changes in their self-concept, relationships, and overall well-being.

9. Termination and relapse prevention: At the end of treatment, therapists and clients review progress made, discuss any remaining concerns, and develop a plan to maintain gains and prevent future relapses.

Korean traditional medicine (KTM) is a system of medicine that has been practiced in Korea for thousands of years. It is also known as Hanbang medicine or Han-ui. KTM is based on the principles of Daoism and the concept of Yin and Yang, and it emphasizes the balance and harmony between the body, mind, and environment.

Korean traditional medicine includes a variety of treatments such as acupuncture, herbal medicine, moxibustion, cupping, and dietary therapy. The use of herbs is a major component of KTM, with thousands of different herbs being used to treat various health conditions. These herbs can be taken in the form of teas, powders, pills, or decoctions.

Acupuncture is also an important part of KTM and involves the insertion of fine needles into specific points on the body to stimulate the flow of Qi (vital energy) and restore balance. Moxibustion involves burning herbs near the skin to warm the area and promote healing, while cupping uses suction cups to increase circulation and relieve pain.

Korean traditional medicine places great emphasis on prevention and encourages individuals to maintain a healthy lifestyle through proper diet, exercise, stress management, and other self-care practices. Practitioners of KTM undergo rigorous training and must pass national exams in order to become licensed.

Nervous system trauma, also known as neurotrauma, refers to damage or injury to the nervous system, including the brain and spinal cord. This type of trauma can result from various causes, such as vehicular accidents, sports injuries, falls, violence, or penetrating traumas. Nervous system trauma can lead to temporary or permanent impairments in sensory, motor, or cognitive functions, depending on the severity and location of the injury.

Traumatic brain injury (TBI) is a common form of nervous system trauma that occurs when an external force causes brain dysfunction. TBIs can be classified as mild, moderate, or severe, based on factors such as loss of consciousness, memory loss, and neurological deficits. Mild TBIs, also known as concussions, may not cause long-term damage but still require medical attention to ensure proper healing and prevent further complications.

Spinal cord injuries (SCI) are another form of nervous system trauma that can have severe consequences. SCI occurs when the spinal cord is damaged due to a sudden, traumatic blow or cut, causing loss of motor function, sensation, or autonomic function below the level of injury. The severity and location of the injury determine the extent of impairment, which can range from partial to complete paralysis.

Immediate medical intervention is crucial in cases of nervous system trauma to minimize secondary damage, prevent complications, and optimize recovery outcomes. Treatment options may include surgery, medication, rehabilitation, or a combination of these approaches.

However, many people with apraxia are no longer able to be independent. Those with limb-kinetic and/or gait apraxia should ... Gait apraxia is the loss of ability to have normal function of the lower limbs such as walking. This is not due to loss of ... Nadeau SE (2007). "Gait apraxia: further clues to localization". Eur. Neurol. 58 (3): 142-5. doi:10.1159/000104714. PMID ... Also, apraxia possibly may be caused by lesions in other areas of the brain. Ideomotor apraxia is typically due to a decrease ...
Urinary incontinence and gait apraxia may also occur. Occlusive damage to Heubner's artery may also present with contralateral ...
"Walking difficulties in patients with Alzheimer's disease might originate from gait apraxia" (PDF). Journal of Neurology, ... "Do Alzheimer's and Lewy body disease have discrete pathological signatures of gait?". Alzheimer's & Dementia. 15 (10): 1367- ...
... , or frontal ataxia, is a gait apraxia found in patients with bilateral frontal lobe disorders. It is ... The gait is broad-based with short steps with a tendency to fall backwards. It was originally described in patients with ... Bruns apraxia can be distinguished from Parkinsonian ataxia and cerebellar ataxia in a number of ways. Patients typically ... This indicates that cerebellar function is intact and that the presented symptoms of Bruns apraxia are due to damage located ...
Parkinsonism Alien hand syndrome Apraxia (ideomotor apraxia and limb-kinetic apraxia) Aphasia The presence of parkinsonism as a ... In another study of 36, over half had a useless/alien arm and 27% had a gait impediment[21]. From this, we can see why CBD is ... Ideomotor apraxia (IMA), although clearly present in CBD, often manifests atypically due to the additional presence of ... Some individuals with CBD exhibit limb-kinetic apraxia, which involves dysfunction of more fine motor movements often performed ...
... gait disorders, neurologic MeSH C23.888.592.413.400 - gait apraxia MeSH C23.888.592.413.450 - gait ataxia MeSH C23.888.592.544 ... apraxias MeSH C23.888.592.604.882.350.200 - apraxia, ideomotor MeSH C23.888.592.604.882.350.600 - gait apraxia MeSH C23.888. ... gait ataxia MeSH C23.888.592.350.110 - athetosis MeSH C23.888.592.350.200 - catalepsy MeSH C23.888.592.350.250 - chorea MeSH ... meningism MeSH C23.888.592.604 - neurobehavioral manifestations MeSH C23.888.592.604.080 - apraxias MeSH C23.888.592.604.115 - ...
... apraxias MeSH F01.700.080.500 - gait apraxia MeSH F01.700.165 - catatonia MeSH F01.700.250 - confusion MeSH F01.700.250.500 - ... apraxias MeSH F01.700.875.350.200 - apraxia, ideomotor MeSH F01.700.875.700 - psychomotor agitation MeSH F01.752.049 - ...
... apraxias MeSH C10.597.606.881.350.200 - apraxia, ideomotor MeSH C10.597.606.881.350.600 - gait apraxia MeSH C10.597.606.881.700 ... gait apraxia MeSH C10.597.404.450 - gait ataxia MeSH C10.597.606.115 - catatonia MeSH C10.597.606.150 - communication disorders ... gait ataxia MeSH C10.597.350.110 - athetosis MeSH C10.597.350.200 - catalepsy MeSH C10.597.350.250 - chorea MeSH C10.597. ...
Movement Tremor Apraxia Dystonia Gait abnormality Clumsiness Emotional Difficulty in inhibiting emotions, anger, excitement ...
... due to involvement of leg part of the motor cortex Cortical sensory loss in the opposite foot and leg Gait apraxia (impairment ... of gait and stance) Abulia, akinetic mutism, slowness and lack of spontaneity Urinary incontinence which usually occurs with ...
... altered walking gait altered movement coordination vertigo and or disequilibrium Loss of consciousness, headache, and vomiting ... apraxia (altered voluntary movements) visual field defect memory deficits (involvement of temporal lobe) hemineglect ( ...
As a result of this gait impairment, falling is a concern in patients with ataxia. Truncal ataxia affects the muscles closer to ... Phenytoin Adrenoleukodystrophy Ataxia oculomotor apraxia type 1 Branchial myoclonus Christianson syndrome Dandy-Walker syndrome ... Truncal ataxia (or trunk ataxia) is a wide-based "drunken sailor" gait characterised by uncertain starts and stops, lateral ... It is involved in gait stability. Truncal ataxia is different from appendicular ataxia. Appendicular ataxia affects the ...
Yaguramaki, N., & Kimura, T. (2002). Acquirement of stability and mobility in infant gait. Gait & Posture, 16(1), 69. Fortney, ... Apraxia Developmental coordination disorder Fine motor skill Santrock, John W. (2008). A Topical Approach to Lifespan ... They also show a synchronized use of hip and shoulder while they are walking, which is different from a mature gait pattern ... Oswalt) Significant changes in gait patterns are also observed in the second year. Infants in the second year have a discordant ...
Classification of the gait pattern according to the Amsterdam Gait Classification: In gait type 1, the knee angle is normal and ... Related conditions can include apraxia, sensory impairments, urinary incontinence, fecal incontinence, or behavioural disorders ... According to the Amsterdam Gait Classification, five gait types are described. To assess the gait pattern, the patient is ... With gait type 5, the knee angle is bent and the foot contact is complete. Gait types 5 is also known as crouch gait. Because ...
These include:[citation needed] Ataxia-oculomotor apraxia type 1 (AOA1) Ataxia-oculomotor apraxia type 2 (AOA2 also known as ... The diagnosis of A-T may not be made until the preschool years when the neurologic symptoms of impaired gait, hand coordination ... Ataxia-oculomotor apraxia type 2 (AOA2) is an autosomal recessive disorder also similar to A-T in manifesting increasing ... Cogan's oculomotor apraxia is generally an isolated problem, or may be associated with broader developmental delay.[citation ...
Gait aids (such as a cane or walker) can be provided to decrease the risk of falls associated with impairment of balance or ... Ataxic cerebral palsy Locomotor ataxia Bruns apraxia National Ataxia Foundation "Dystaxia Definition & Meaning". Dictionary.com ... Friedreich's ataxia has gait abnormality as the most commonly presented symptom. Dystaxia is a mild degree of ataxia. The term ... As a result of this gait impairment, falling is a concern in patients with ataxia. Studies examining falls in this population ...
The symptoms are broad, but consistently involve gait and limb ataxia, dysarthria and loss of lower limb reflexes. There is ... Other diagnoses might include Charcot-Marie-Tooth types 1 and 2, ataxia with vitamin E deficiency, ataxia-oculomotor apraxia ... The disease is progressive, with increasing staggering or stumbling gait and frequent falling. By the third decade, affected ... March 2017). "A wearable proprioceptive stabilizer for rehabilitation of limb and gait ataxia in hereditary cerebellar ataxias ...
Such devices may include a cane, crutches, walker, or wheelchair for those with impaired gait. Other devices are available to ... Moreira, Maria-Ceu; Koenig, Michel (December 8, 2011). Ataxia with Oculomotor Apraxia Type 2. University of Washington, Seattle ... In general, physical therapy emphasises postural balance and gait training for ataxia patients. General conditioning such as ... gait, and activities of daily living. Some level of improvement was shown to be maintained 24 weeks post-treatment. Speech ...
Since apraxia of speech is said to be due to weak feedforward programs and high dependence on auditory feedback, auditory ... Auditory feedback in the form of periodic audio signals was found to have a significant improvement on the gait of patients, ... Jacks, A.; Haley, K.L (2015). "Auditory Masking Effects on Speech Fluency in Apraxia of Speech and Aphasia: Comparison to ... It is posited that individuals with apraxia of speech have weak feedforward programs, which results in the disfluencies of ...
Individuals are often able to identify others through alternate characteristics, such as, voice, gait, context or unique facial ... Greene, J. D. W (2005). "Apraxia, agnosias, and higher visual function abnormalities". Journal of Neurology, Neurosurgery, and ...
There is some evidence that rhythmic auditory stimulation is beneficial in gait rehabilitation following a brain injury. Music ... Symptoms: Anhedonia Apraxia Aphasia Amnesia Behavioral manifestations: Adynamia Disinhibition Emotional flooding Perseveration ... therapy may assist patients to improve gait, arm swing while walking, communication, and quality of life after experiencing a ...
Ocular problems related to the disorder include strabismus, nystagmus, retinitis, disc pallor, and oculomotor apraxia. Over ... and improvement in coordination and gait. During prolonged periods of fasting, ketone bodies serve as the primary energy source ... Neonatal problems EEG abnormalities Psychoses MRI or X-ray computed tomography abnormalities Oculomotor apraxia Microcephaly ...
Apraxia, motor problems, and seizures are prominent during this stage. However, there may be improvement in behavior, with less ... Ruling in Decreased or loss of use of fine motor skills Decreased or loss of verbal speech Abnormalities during gait Repetitive ... because of difficulty walking or malnutrition caused by difficulty swallowing hypotonia delayed or absent ability to walk gait/ ...
Areas of interest concerning gait during rehabilitation programs focus on improving gait speed, the base of support, stride ... Some key symptoms: Corticobasal syndrome - levodopa-resistance, myoclonus, dystonia, corticosensory loss, apraxia, and non- ... As for gait and addressing the challenges associated with the disease such as hypokinesia, shuffling, and decreased arm swing, ... Other recognized motor signs and symptoms include gait and posture disturbances such as festination (rapid shuffling steps and ...
However, many people with apraxia are no longer able to be independent. Those with limb-kinetic and/or gait apraxia should ... Gait apraxia is the loss of ability to have normal function of the lower limbs such as walking. This is not due to loss of ... Nadeau SE (2007). "Gait apraxia: further clues to localization". Eur. Neurol. 58 (3): 142-5. doi:10.1159/000104714. PMID ... Also, apraxia possibly may be caused by lesions in other areas of the brain. Ideomotor apraxia is typically due to a decrease ...
... gait refers to how a person walks. An abnormal gait might be caused by an underlying physical condition, disease or injury. ... ClinicalTrials.gov: Gait Apraxia (National Institutes of Health) * ClinicalTrials.gov: Gait Ataxia (National Institutes of ... The pattern of how you walk is called your gait. Many different diseases and conditions can affect your gait and lead to ... Gait or Walking Problems (National Multiple Sclerosis Society) - PDF * Hallux Rigidus (American College of Foot and Ankle ...
... is a neurological condition with gait apraxia signs from its early manifestation. Ventriculoperitoneal shunt (VPS) is a ... is aimed to test the hypothesis that wearable motion sensors provide valid measures to manage iNPH patients with gait apraxia. ... 21 instrumental variables described gait and postural transitions from TUG and 18mW recordings. Furthermore, participants were ... and instrumental variables showed statistically significant improvements that reflect a reduction of apraxic features of gait. ...
Apraxia of gait- or apraxia of postural transitions? Dale ML, Curtze C, Nutt JG. Dale ML, et al. Among authors: nutt jg. ... Higher level gait disorders. Thompson PD, Nutt JG. Thompson PD, et al. Among authors: nutt jg. J Neural Transm (Vienna). 2007; ... Freezing of gait: moving forward on a mysterious clinical phenomenon. Nutt JG, Bloem BR, Giladi N, Hallett M, Horak FB, ... Discussion of Research Priorities for Gait Disorders in Parkinsons Disease. Bohnen NI, Costa RM, Dauer WT, Factor SA, Giladi N ...
Gait Apraxia and Hakims Disease: A Historical Review. Previous Article in Special Issue. An Observational Study on Chronic ...
The classic Hakim triad of symptoms includes gait apraxia, incontinence, and dementia. Headache is not a typical symptom in NPH ... Gait disturbance: Usually the first symptom and may precede other symptoms by months or years; magnetic gait is used to ... Variable difficulty in walking: May have mild imbalance to inability to walk or to stand; the classic gait impairment consists ... a hallmark of the gait impairment of Parkinson disease) ...
Speech & Language: Aphasia, Apraxia, Dysarthria, Voice Disorders. *Sensorimotor: Upper Extremity, Lower Extremity, Gait ...
Risk factors for the syndrome of ventricular enlargement with gait apraxia. J. Neurol. Neurosurg. Psych., 52: 847-852.. PubMed ...
For example, lesions in the anterior cerebral artery involve symptoms of urinary incontinence, apraxia of gait and motor mutism ...
Focal signs tend to be related to posterior brain (eg, occipital lobes). Gait abnormalities occur in up to 65% patients, and ... Focal neurologic signs include aphasia, hemiparesis, ataxia, cortical blindness, limb apraxia, brainstem symptoms and, less ...
... between age of 1 and 4 years the gait is punctuated by significant truncal ataxia and apraxia demonstrating a broad-based, ... For those who do not walk, weight-bearing should be an essential part of therapy, either standing frames or gait-trainers and ... Anxiety is a major problem in many aspects of RTT and will certainly lessen security in maintaining independent gait or even in ... have independent gait. The majority of those no longer walking independently are able to walk with assistance. In some this ...
A man in early adolescence, born out of consanguineous marriage, presented with progressive gait unsteadiness and recurrent ... On examination, he had bulbar conjunctival telangiectasia (figure 1), oculomotor apraxia, bilateral cerebellar signs and ... Abnormal eye movements such as oculomotor apraxia, nystagmus, accommodation abnormalities and hypometric saccades may be ...
Ataxia and apraxia are often confused for each other, but they are two very different neurological symptoms. Ataxia is a ... Difference between ataxia and apraxia Ataxia vs apraxia Neurological lesions have always been complicated as understanding the ... Cerebellar ataxia presents itself as disturbances in gait, difficulty in balancing, disturbances in walking and other motor ... The types of apraxia are ideomotor apraxia, conceptual apraxia, speech apraxia and constructional apraxia. Ideomotor apraxia ...
ideomotor apraxia in persons with AD at all stages of the disease ( Sheridan & Hausdorff, 2007 ). In particular, persons with ... AD have been shown to have shorter step length, slower gait speed and stepping frequency, greater step-to-step variability, and ...
Speech Apraxia. Smith-Magenis Syndrome. Spina Bifida. Temperature Instability. Torticollis. Traumatic Brain Injury. Trisomy P ... Crouch Gait. Developmental Delays. Dravet Syndrome. Encephalitis. Epilepsy. Fetal Alcohol Spectrum Disorder. Fine Motor Delays ... Apraxia. Arthrogryposis. Aspergers Disorder. Asthma. Ataxia. Autism. Bilateral Club Feet. Bipolar Disorder. Bone Disorder. ... Ocular Motor Apraxia. Osteogenesis Imperfecta. Osteoporosis. Parkinsons. Patent Ductess Arteriosis. PDD-NOS. Pertussis. ...
Gait apraxia evaluation in normal pressure hydrocephalus using inertial sensors. Clinical correlates, ventriculoperitoneal ... Idiopathic normal pressure hydrocephalus (iNPH) is a neurological condition with gait apraxia signs from its early ...
APRAXIA DE LA MARCHA. GAIT APRAXIA. APRAXIA DA MARCHA. APRAXIA IDEOMOTORA. APRAXIA, IDEOMOTOR. APRAXIA IDEOMOTORA. ... GAIT DISORDERS, NEUROLOGIC. TRANSTORNOS NEUROLÓGICOS DA MARCHA. TRASTORNOS PARKINSONIANOS. PARKINSONIAN DISORDERS. TRANSTORNOS ...
APRAXIA DE LA MARCHA. GAIT APRAXIA. APRAXIA DA MARCHA. APRAXIA IDEOMOTORA. APRAXIA, IDEOMOTOR. APRAXIA IDEOMOTORA. ... GAIT DISORDERS, NEUROLOGIC. TRANSTORNOS NEUROLÓGICOS DA MARCHA. TRASTORNOS PARKINSONIANOS. PARKINSONIAN DISORDERS. TRANSTORNOS ...
APRAXIA DE LA MARCHA. GAIT APRAXIA. APRAXIA DA MARCHA. APRAXIA IDEOMOTORA. APRAXIA, IDEOMOTOR. APRAXIA IDEOMOTORA. ... GAIT DISORDERS, NEUROLOGIC. TRANSTORNOS NEUROLÓGICOS DA MARCHA. TRASTORNOS PARKINSONIANOS. PARKINSONIAN DISORDERS. TRANSTORNOS ...
GAIT APRAXIA APRAXIA DE LA MARCHA APRAXIA DA MARCHA GAIT ATAXIA ATAXIA DE LA MARCHA MARCHA ATÁXICA ... GAIT DISORDERS, NEUROLOGIC TRASTORNOS NEUROLOGICOS DE LA MARCHA TRANSTORNOS NEUROLÓGICOS DA MARCHA ...
GAIT APRAXIA APRAXIA DE LA MARCHA APRAXIA DA MARCHA GAIT ATAXIA ATAXIA DE LA MARCHA MARCHA ATÁXICA ... GAIT DISORDERS, NEUROLOGIC TRASTORNOS NEUROLOGICOS DE LA MARCHA TRANSTORNOS NEUROLÓGICOS DA MARCHA ...
APRAXIA DA MARCHA GAIT APRAXIA APRAXIA DE LA MARCHA APRAXIA IDEOMOTORA APRAXIA, IDEOMOTOR APRAXIA IDEOMOTORA ... GAIT DISORDERS, NEUROLOGIC TRASTORNOS NEUROLOGICOS DE LA MARCHA TRANSTORNOS PARKINSONIANOS PARKINSONIAN DISORDERS TRASTORNOS ...
APRAXIA DE LA MARCHA. GAIT APRAXIA. APRAXIA DA MARCHA. APRAXIA IDEOMOTORA. APRAXIA, IDEOMOTOR. APRAXIA IDEOMOTORA. ... GAIT DISORDERS, NEUROLOGIC. TRANSTORNOS NEUROLÓGICOS DA MARCHA. TRASTORNOS PARKINSONIANOS. PARKINSONIAN DISORDERS. TRANSTORNOS ...
GAIT APRAXIA APRAXIA DE LA MARCHA APRAXIA DA MARCHA GAIT ATAXIA ATAXIA DE LA MARCHA MARCHA ATÁXICA ... GAIT DISORDERS, NEUROLOGIC TRASTORNOS NEUROLOGICOS DE LA MARCHA TRANSTORNOS NEUROLÓGICOS DA MARCHA ...
APRAXIA DA MARCHA GAIT APRAXIA APRAXIA DE LA MARCHA APRAXIA IDEOMOTORA APRAXIA, IDEOMOTOR APRAXIA IDEOMOTORA ... GAIT DISORDERS, NEUROLOGIC TRASTORNOS NEUROLOGICOS DE LA MARCHA TRANSTORNOS PARKINSONIANOS PARKINSONIAN DISORDERS TRASTORNOS ...
GAIT APRAXIA APRAXIA DE LA MARCHA APRAXIA DA MARCHA GAIT ATAXIA ATAXIA DE LA MARCHA MARCHA ATÁXICA ... GAIT DISORDERS, NEUROLOGIC TRASTORNOS NEUROLOGICOS DE LA MARCHA TRANSTORNOS NEUROLÓGICOS DA MARCHA ...
GAIT APRAXIA APRAXIA DE LA MARCHA APRAXIA DA MARCHA GAIT ATAXIA ATAXIA DE LA MARCHA MARCHA ATÁXICA ... GAIT DISORDERS, NEUROLOGIC TRASTORNOS NEUROLOGICOS DE LA MARCHA TRANSTORNOS NEUROLÓGICOS DA MARCHA ...
APRAXIA DA MARCHA GAIT APRAXIA APRAXIA DE LA MARCHA APRAXIA IDEOMOTORA APRAXIA, IDEOMOTOR APRAXIA IDEOMOTORA ... GAIT DISORDERS, NEUROLOGIC TRASTORNOS NEUROLOGICOS DE LA MARCHA TRANSTORNOS PARKINSONIANOS PARKINSONIAN DISORDERS TRASTORNOS ...
  • Ataxia and apraxia are often confused for each other, but they are two very different neurological symptoms. (differencebetween.net)
  • Ataxia is due to lesions in the cerebellum but apraxia is due to lesions in the cerebrum. (differencebetween.net)
  • Ataxia is seen as 'muscles giving way' but apraxia is the result of a lack of a motor impulse in spite of having intact muscular power and the ability to do it. (differencebetween.net)
  • Ataxia is a sign of some cerebellar condition, it is the result of a lesion in the cerebellum but apraxia is a clinical condition itself. (differencebetween.net)
  • While ataxia is loss of sensory and motor functions, apraxia is lack of just the motor functions i.e. muscular movements. (differencebetween.net)
  • Cerebellar ataxia presents itself as disturbances in gait, difficulty in balancing, disturbances in walking and other motor disturbances like tremors and difficulty in taking a turn while walking. (differencebetween.net)
  • Ataxia is loss of controlled and coordinated muscle movements due to muscle weakness whereas apraxia is inability to carry out purposeful movements despite proper coordination and muscle power. (differencebetween.net)
  • In ataxia, a person has inability due to defect in the nervous pathways crossing over at cerebellum, but in apraxia all the complex integrated movements are difficult to be performed. (differencebetween.net)
  • See also Ataxia with Oculomotor Apraxia 1 ( 208920 ) with hypoalbuminemia, Ataxia with Oculomotor Apraxia 2 ( 606002 ) (also known as Spinocerebellar Ataxia, Autosomal Recessive 1 or SCAR1), and Ataxia with Oculomotor Apraxia 4 ( 616267 ). (arizona.edu)
  • EBF3 neurodevelopmental disorder (EBF3-NDD) is associated with developmental delay (DD) / intellectual disability (ID), speech delay, gait or truncal ataxia, hypotonia, behavioral problems, and facial dysmorphism. (beds.ac.uk)
  • Ideational apraxia has been observed in patients with lesions in the dominant hemisphere near areas associated with aphasia, but more research is needed on ideational apraxia due to brain lesions. (wikipedia.org)
  • Improvements noted in speech, aphasia, apraxia and walking within minutes after treatment at the INR in Boca Raton, Florida. (tobinick.com)
  • Apraxia of speech is often present along with another speech disorder called aphasia. (diseasesdic.com)
  • These signs and symptoms are apraxia, alien hand syndrome, aphasia and parkinsonism. (allhealthsite.com)
  • Buccofacial or orofacial apraxia, the most common type of apraxia, is the inability to carry out facial movements on demand. (wikipedia.org)
  • An example of this type of apraxia is that a person puts vegetables in the pot first and then the oil needed for cooking. (differencebetween.net)
  • People with this type of apraxia have difficulty making eye movements on command. (diseasesdic.com)
  • The multiple types of apraxia are categorized by the specific ability and/or body part affected. (wikipedia.org)
  • The several types of apraxia include: Apraxia of speech (AOS) is having difficulty planning and coordinating the movements necessary for speech (e.g. potato=totapo, topato). (wikipedia.org)
  • The types of apraxia are ideomotor apraxia, conceptual apraxia, speech apraxia and constructional apraxia. (differencebetween.net)
  • One form is orofacial apraxia. (diseasesdic.com)
  • People with orofacial apraxia are unable to voluntarily perform certain movements involving facial muscles. (diseasesdic.com)
  • Ideomotor apraxia is having deficits in the ability to plan or complete motor actions that rely on semantic memory. (wikipedia.org)
  • Ideomotor apraxia is typically due to a decrease in blood flow to the dominant hemisphere of the brain and particularly the parietal and premotor areas. (wikipedia.org)
  • Ideomotor apraxia manifests as an inability to plan or complete voluntary actions like closing the buttons of the shirt, etc. (differencebetween.net)
  • People with ideomotor apraxia are unable to follow a verbal command to copy the movements of others or follow suggestions for movements. (diseasesdic.com)
  • This form of apraxia is similar to ideomotor apraxia. (diseasesdic.com)
  • Ideational/conceptual apraxia is having an inability to conceptualize a task and impaired ability to complete multistep actions. (wikipedia.org)
  • Conceptual apraxia is seen as not having the ability to think over the steps required to perform some action. (differencebetween.net)
  • People with conceptual apraxia are also unable to perform tasks that involve more than one step. (diseasesdic.com)
  • Apraxia is a motor disorder caused by damage to the brain (specifically the posterior parietal cortex or corpus callosum), which causes difficulty with motor planning to perform tasks or movements. (wikipedia.org)
  • Limb-kinetic apraxia is having the inability to perform precise, voluntary movements of extremities. (wikipedia.org)
  • Oculomotor apraxia is having difficulty moving the eye on command, especially with saccade movements that direct the gaze to targets. (wikipedia.org)
  • Abnormal eye movements such as oculomotor apraxia, nystagmus, accommodation abnormalities and hypometric saccades may be associated with A-T. Mutation of the ATM gene responsible for coordinating the signalling pathways in response to genotoxicity leads to A-T. 6 The importance of vigilant ophthalmic examination in the diagnosis of hereditary neurodegenerative disorders is hereby emphasised. (bmj.com)
  • Apraxia, on the other hand, is the inability to execute complex, purposeful movements which have already been learnt by the person. (differencebetween.net)
  • An ataxic gait is characterized by imbalance, and abnormal, uncoordinated movements. (nursepartners.org)
  • Apraxia (or dyspraxia if it is mild) is a neurological disorder characterized by loss of the ability to carry out purposeful movements and gestures, despite having the desire and the physical ability to perform them. (mmclt.org)
  • People with limb-kinetic apraxia are unable to use a finger, arm, or leg to make precise and coordinated movements. (diseasesdic.com)
  • People with buccofacial apraxia, or facial-oral apraxia, are unable to make movements with the face and lips on command. (diseasesdic.com)
  • People with verbal or oral apraxia find it challenging to make the movements necessary for speech. (diseasesdic.com)
  • A person with apraxia is unable to put together the correct muscle movements. (diseasesdic.com)
  • In addition, apraxia may also occur and affect fine motor movements normally performed by the hands as well as the digits. (allhealthsite.com)
  • This form of apraxia consists of an inability to select and carry out an appropriate motor program. (wikipedia.org)
  • Another form of apraxia affects a person's ability to intentionally move arms and legs. (diseasesdic.com)
  • The cause of apraxia is a lesion in the cerebrum. (differencebetween.net)
  • Depending on the cause of apraxia, a number of other brain or nervous system problems may be present. (diseasesdic.com)
  • This dedicated unit consists of a team of professionals who are skilled in treating neuromotor disorders such as cerebral palsy, apraxia etc. (nish.ac.in)
  • For the first time in 1977 Miller-Fisher thoroughly described iNPH gait alterations using the term "gait apraxia" [ 5 ]. (biomedcentral.com)
  • Considering that the classical definition of apraxia of gait is " loss of ability to properly use lower limbs in the act of walking which cannot be accounted for by demonstrable sensory impairment or motor weakness" [ 6 ], also today this term represents a broadly accepted definition of iNPH gait alterations in literature. (biomedcentral.com)
  • The most common description of iNPH gait is "shuffling," "magnetic," and "wide-based. (capsulehealth.one)
  • Childhood apraxia of speech, acquired apraxia of speech, and non-verbal oral apraxia (NVOA) are treated by speech therapy. (differencebetween.net)
  • The localization of lesions in areas of the frontal and temporal lobes would provide explanation for the difficulty in motor planning seen in ideational apraxia, as well as its difficulty to distinguish it from certain aphasias. (wikipedia.org)
  • People with ideational apraxia are unable to plan a particular movement. (diseasesdic.com)
  • Constructional apraxia is the inability to draw, construct, or copy simple configurations, such as intersecting shapes. (wikipedia.org)
  • Constructional apraxia is often caused by lesions of the inferior nondominant parietal lobe, and can be caused by brain injury, illness, tumor, or other condition that can result in a brain lesion. (wikipedia.org)
  • People with constructional apraxia are unable to copy, draw, or construct basic diagrams or figures. (diseasesdic.com)
  • Apraxia is another condition associated with dementia and the ability to walk. (nursepartners.org)
  • Progressive gait impairments, urinary incontinence, and cognitive deficits define the clinical triad that highlights its onset [ 1 ]. (biomedcentral.com)
  • Potentially reversible syndrome characterized by enlarged cerebral ventricles (ventriculomegaly), cognitive impairment, gait apraxia and urinary incontinence. (capsulehealth.one)
  • Idiopathic normal pressure hydrocephalus (iNPH) is a neurological condition with gait apraxia signs from its early manifestation. (biomedcentral.com)
  • Apraxia is a poorly understood neurological condition. (diseasesdic.com)
  • Affected individuals have delayed motor development due to severe distal motor and sensory impairment, resulting in difficulties in gait. (nih.gov)
  • I am a disabled man with proven brain damage, apraxia, limited mobility, speech and gait impairment. (justia.com)
  • For example, a person affected by limb apraxia may have difficulty waving hello, tying shoes, or typing on a computer. (wikipedia.org)
  • Apraxia is a condition wherein an individual loses the ability to perform tasks that are normally familiar and are previously done without difficulty. (allhealthsite.com)
  • Onset of gait instability occurs in the second decade of life with dysmetria and frequent falls. (arizona.edu)
  • The occurrence of gait abnormality before the onset of cognitive decline has been reported to predict a better prognosis after shunting. (capsulehealth.one)
  • Impairments of gait and balance are typically the first symptoms to be noticed and may be very mild at the outset. (capsulehealth.one)
  • Generally, gait disturbance plus one additional feature is required to consider the diagnosis. (capsulehealth.one)
  • With disease progression, the patient's gait deteriorates finally becoming broad-based, slow, short-stepped, and glue-footed (a gait disturbance of the astasia-abasia type). (capsulehealth.one)
  • Speech apraxia is seen in both adults and children. (differencebetween.net)
  • Speech Apraxia. (ossweb.com)
  • With apraxia of speech, a person finds it difficult or impossible to move his or her mouth and tongue to speak. (diseasesdic.com)
  • The physiotherapist provides training to improve motor skills, gait, balance, coordination and endurance of movement. (nish.ac.in)
  • Apraxia is an acquired disorder of motor nerves due to inability to understand commands. (differencebetween.net)
  • 21 instrumental variables described gait and postural transitions from TUG and 18mW recordings. (biomedcentral.com)
  • For this reason, the correct definition of gait alterations and their clinical and quantitative description, together with a differential diagnosis from similar motor conditions (iNPH mimics), represent critical challenges to address the syndrome in its early phase [ 3 ]. (biomedcentral.com)
  • The gait and cognitive disturbances of iNPH are thought to be of periventricular/subcortical/frontal origin. (capsulehealth.one)
  • For example, apraxia from dementias tends to worsen over time. (mmclt.org)
  • After VPS, most clinical and instrumental variables showed statistically significant improvements that reflect a reduction of apraxic features of gait. (biomedcentral.com)
  • Freezing of gait: moving forward on a mysterious clinical phenomenon. (nih.gov)
  • Also, apraxia possibly may be caused by lesions in other areas of the brain. (wikipedia.org)
  • A man in early adolescence, born out of consanguineous marriage, presented with progressive gait unsteadiness and recurrent sinopulmonary and soft tissue infections for 5 years. (bmj.com)
  • Many different diseases and conditions can affect your gait and lead to problems with walking. (medlineplus.gov)
  • Apraxia is most often due to a lesion located in the dominant (usually left) hemisphere of the brain, typically in the frontal and parietal lobes. (wikipedia.org)
  • Gait apraxia is the loss of ability to have normal function of the lower limbs such as walking. (wikipedia.org)
  • It is not uncommon to see a person go from a slow, cautious gait, to a normal walking pattern simply by taking up a cane. (nursepartners.org)
  • Gait apraxia evaluation in normal pressure hydrocephalus using inertial sensors. (biomedcentral.com)