A family of RNA viruses, many of which cause disease in humans and domestic animals. There are three genera FLAVIVIRUS; PESTIVIRUS; and HEPACIVIRUS, as well as several unassigned species.
Infections with viruses of the family FLAVIVIRIDAE.
A genus of FLAVIVIRIDAE containing several subgroups and many species. Most are arboviruses transmitted by mosquitoes or ticks. The type species is YELLOW FEVER VIRUS.
A group of viruses in the genus PESTIVIRUS, causing diarrhea, fever, oral ulcerations, hemorrhagic syndrome, and various necrotic lesions among cattle and other domestic animals. The two species (genotypes), BVDV-1 and BVDV-2 , exhibit antigenic and pathological differences. The historical designation, BVDV, consisted of both (then unrecognized) genotypes.
A species of virus (unassigned to a genus) in the family FLAVIVIRIDAE, that have been identified in at least six species of New World monkeys. They do not cause HEPATITIS in the host or other susceptible species.
Infections with viruses of the genus FLAVIVIRUS, family FLAVIVIRIDAE.
A genus of FLAVIVIRIDAE, also known as mucosal disease virus group, which is not arthropod-borne. Transmission is by direct and indirect contact, and by transplacental and congenital transmission. Species include BORDER DISEASE VIRUS, bovine viral diarrhea virus (DIARRHEA VIRUS, BOVINE VIRAL), and CLASSICAL SWINE FEVER VIRUS.
A genus in the subfamily CALLITRICHINAE consisting of 12 species and found in Panama as well as South America. Species seen most frequently in the literature are S. oedipus (cotton-top marmoset), S. nigricollis, and S. fusicollis.
A species of FLAVIVIRUS, one of the Japanese encephalitis virus group (ENCEPHALITIS VIRUSES, JAPANESE). It can infect birds and mammals. In humans, it is seen most frequently in Africa, Asia, and Europe presenting as a silent infection or undifferentiated fever (WEST NILE FEVER). The virus appeared in North America for the first time in 1999. It is transmitted mainly by CULEX spp mosquitoes which feed primarily on birds, but it can also be carried by the Asian Tiger mosquito, AEDES albopictus, which feeds mainly on mammals.
A species of FLAVIVIRUS, one of the Japanese encephalitis virus group (ENCEPHALITIS VIRUSES, JAPANESE), which is the etiologic agent of ST. LOUIS ENCEPHALITIS in the United States, the Caribbean, and Central and South America.
Proteins encoded by a VIRAL GENOME that are produced in the organisms they infect, but not packaged into the VIRUS PARTICLES. Some of these proteins may play roles within the infected cell during VIRUS REPLICATION or act in regulation of virus replication or VIRUS ASSEMBLY.
A species of virus (unassigned to a genus) in the family FLAVIVIRIDAE. It is genetically heterogeneous, of human origin, and transmitted by blood or blood products. Despite its alternate name (Hepatitis G virus), its pathogenicity remains controversial.
A species of the PESTIVIRUS genus causing exceedingly contagious and fatal hemorrhagic disease of swine.
An enzyme which catalyzes the hydrolysis of nucleoside triphosphates to nucleoside diphosphates. It may also catalyze the hydrolysis of nucleotide triphosphates, diphosphates, thiamine diphosphates and FAD. The nucleoside triphosphate phosphohydrolases I and II are subtypes of the enzyme which are found mostly in viruses.
A viral encephalitis caused by the St. Louis encephalitis virus (ENCEPHALITIS VIRUS, ST. LOUIS), a FLAVIVIRUS. It is transmitted to humans and other vertebrates primarily by mosquitoes of the genus CULEX. The primary animal vectors are wild birds and the disorder is endemic to the midwestern and southeastern United States. Infections may be limited to an influenza-like illness or present as an ASEPTIC MENINGITIS or ENCEPHALITIS. Clinical manifestations of the encephalitic presentation may include SEIZURES, lethargy, MYOCLONUS, focal neurologic signs, COMA, and DEATH. (From Adams et al., Principles of Neurology, 6th ed, p750)
A genus of mosquitoes (CULICIDAE) commonly found in tropical regions. Species of this genus are vectors for ST. LOUIS ENCEPHALITIS as well as many other diseases of man and domestic and wild animals.
A species of the genus FLAVIVIRUS which causes an acute febrile and sometimes hemorrhagic disease in man. Dengue is mosquito-borne and four serotypes are known.
A family of proteins that promote unwinding of RNA during splicing and translation.
INFLAMMATION of the LIVER in animals due to viral infection.
Arthropod-borne viruses. A non-taxonomic designation for viruses that can replicate in both vertebrate hosts and arthropod vectors. Included are some members of the following families: ARENAVIRIDAE; BUNYAVIRIDAE; REOVIRIDAE; TOGAVIRIDAE; and FLAVIVIRIDAE. (From Dictionary of Microbiology and Molecular Biology, 2nd ed)
A species of FLAVIVIRUS, one of the Japanese encephalitis virus group (ENCEPHALITIS VIRUSES, JAPANESE), which is the etiological agent of Japanese encephalitis found in Asia, southeast Asia, and the Indian subcontinent.
The complete genetic complement contained in a DNA or RNA molecule in a virus.
The type species of the FLAVIVIRUS genus. Principal vector transmission to humans is by AEDES spp. mosquitoes.
Ribonucleic acid that makes up the genetic material of viruses.
A mosquito-borne viral illness caused by the WEST NILE VIRUS, a FLAVIVIRUS and endemic to regions of Africa, Asia, and Europe. Common clinical features include HEADACHE; FEVER; maculopapular rash; gastrointestinal symptoms; and lymphadenopathy. MENINGITIS; ENCEPHALITIS; and MYELITIS may also occur. The disease may occasionally be fatal or leave survivors with residual neurologic deficits. (From Joynt, Clinical Neurology, 1996, Ch26, p13; Lancet 1998 Sep 5;352(9130):767-71)
Infections caused by arthropod-borne viruses, general or unspecified.
An enzyme that catalyses RNA-template-directed extension of the 3'- end of an RNA strand by one nucleotide at a time, and can initiate a chain de novo. (Enzyme Nomenclature, 1992, p293)
A genus of FLAVIVIRIDAE causing parenterally-transmitted HEPATITIS C which is associated with transfusions and drug abuse. Hepatitis C virus is the type species.
A family of the order DIPTERA that comprises the mosquitoes. The larval stages are aquatic, and the adults can be recognized by the characteristic WINGS, ANIMAL venation, the scales along the wing veins, and the long proboscis. Many species are of particular medical importance.
INFLAMMATION of the LIVER in humans due to infection by VIRUSES. There are several significant types of human viral hepatitis with infection caused by enteric-transmission (HEPATITIS A; HEPATITIS E) or blood transfusion (HEPATITIS B; HEPATITIS C; and HEPATITIS D).
Acute disease of cattle caused by the bovine viral diarrhea viruses (DIARRHEA VIRUSES, BOVINE VIRAL). Often mouth ulcerations are the only sign but fever, diarrhea, drop in milk yield, and loss of appetite are also seen. Severity of clinical disease varies and is strain dependent. Outbreaks are characterized by low morbidity and high mortality.
The interactions between a host and a pathogen, usually resulting in disease.
The relationships of groups of organisms as reflected by their genetic makeup.
The process of intracellular viral multiplication, consisting of the synthesis of PROTEINS; NUCLEIC ACIDS; and sometimes LIPIDS, and their assembly into a new infectious particle.
Insects that transmit infective organisms from one host to another or from an inanimate reservoir to an animate host.
Invertebrates or non-human vertebrates which transmit infective organisms from one host to another.
Descriptions of specific amino acid, carbohydrate, or nucleotide sequences which have appeared in the published literature and/or are deposited in and maintained by databanks such as GENBANK, European Molecular Biology Laboratory (EMBL), National Biomedical Research Foundation (NBRF), or other sequence repositories.
Any member of the group of ENDOPEPTIDASES containing at the active site a serine residue involved in catalysis.
A genus of mosquitoes (CULICIDAE) frequently found in tropical and subtropical regions. YELLOW FEVER and DENGUE are two of the diseases that can be transmitted by species of this genus.
Agents used in the prophylaxis or therapy of VIRUS DISEASES. Some of the ways they may act include preventing viral replication by inhibiting viral DNA polymerase; binding to specific cell-surface receptors and inhibiting viral penetration or uncoating; inhibiting viral protein synthesis; or blocking late stages of virus assembly.
Layers of protein which surround the capsid in animal viruses with tubular nucleocapsids. The envelope consists of an inner layer of lipids and virus specified proteins also called membrane or matrix proteins. The outer layer consists of one or more types of morphological subunits called peplomers which project from the viral envelope; this layer always consists of glycoproteins.
Domesticated bovine animals of the genus Bos, usually kept on a farm or ranch and used for the production of meat or dairy products or for heavy labor.
Established cell cultures that have the potential to propagate indefinitely.
An acute febrile disease transmitted by the bite of AEDES mosquitoes infected with DENGUE VIRUS. It is self-limiting and characterized by fever, myalgia, headache, and rash. SEVERE DENGUE is a more virulent form of dengue.
Proteins found in any species of virus.
The sequence of PURINES and PYRIMIDINES in nucleic acids and polynucleotides. It is also called nucleotide sequence.
The order of amino acids as they occur in a polypeptide chain. This is referred to as the primary structure of proteins. It is of fundamental importance in determining PROTEIN CONFORMATION.
INFLAMMATION of the LIVER in humans caused by HEPATITIS C VIRUS, a single-stranded RNA virus. Its incubation period is 30-90 days. Hepatitis C is transmitted primarily by contaminated blood parenterally, and is often associated with transfusion and intravenous drug abuse. However, in a significant number of cases, the source of hepatitis C infection is unknown.
A multistage process that includes cloning, physical mapping, subcloning, determination of the DNA SEQUENCE, and information analysis.
The spatial arrangement of the atoms of a nucleic acid or polynucleotide that results in its characteristic 3-dimensional shape.
A subfamily in the family MURIDAE, comprising the hamsters. Four of the more common genera are Cricetus, CRICETULUS; MESOCRICETUS; and PHODOPUS.

Carriage of GB virus C/hepatitis G virus RNA is associated with a slower immunologic, virologic, and clinical progression of human immunodeficiency virus disease in coinfected persons. (1/233)

The prevalence of GB virus C (GBV-C) infection is high in human immunodeficiency virus (HIV)-infected persons. However, the long-term consequences of coinfection are unknown. HIV-positive persons with a well-defined duration of infection were screened on the basis of their GBV-C/hepatitis G virus (HGV) RNA status and studied. GBV-C/HGV viremia was observed in 23, who carried the virus over a mean of 7.7 years. All parameters (survival, CDC stage B/C, HIV RNA load, CD4 T cell count) showed significant differences in terms of the cumulative progression rate between persons positive and negative for GBV-C/HGV RNA. When GBV-C/HGV RNA-positive and -unexposed subjects were matched by age, sex, baseline HIV RNA load, and baseline CD4 T cell count, HIV disease progression appeared worse in GBV-C/HGV RNA-negative subjects. The carriage of GBV-C/HGV RNA is associated with a slower progression of HIV disease in coinfected persons.  (+info)

High prevalence of hepatitis G virus (HGV) infections in dialysis staff. (2/233)

BACKGROUND: Patients on renal replacement therapy, haemodialysis (HD), or after kidney transplantation (TX), are known to be at risk of acquiring blood-borne infections (HBV, HCV). GBV-C/Hepatitis G virus (HGV) has been described recently and is considered to cause blood-borne infections. The aim of this study was to analyse the risk for the medical staff of HD and TX patients to acquire HGV infection. METHODS: Eighty-five HD patients and 86 TX recipients were compared with 49 health-care workers and 64 blood donors as controls. The HGV prevalence was determined by RT-PCR and antibodies to E2 protein. RESULTS: A high prevalence of HGV was found in the medical staff (24%) which nearly corresponded to the prevalence of the patients (TX 36%, HD 25%) but not to the controls (9%). In contrast, the prevalence of HCV was low in the medical staff (2%) and controls (0%) but high in HD (13%) and TX (13%). Age and duration of employment in the department did not significantly influence the HGV prevalence in staff. The number of viraemic subjects in staff was high, possibly indicating a more recent infection. CONCLUSION: An occupational risk for HGV exists in medical staff of dialysis and transplant patients. Further routes of transmission than only parenteral may play a role in this setting.  (+info)

Sequence heterogeneity within three different regions of the hepatitis G virus genome. (3/233)

Two sets of primers derived from the 5'-terminal region and the NS5 region of the hepatitis G virus (HGV) genome were used to amplify PCR fragments from serum specimens obtained from different parts of the world. All PCR fragments from the 5'-terminal region (5'-PCR, n = 56) and from the NS5 region (NS5-PCR, n = 85) were sequenced and compared to corresponding published HGV sequences. The range of nucleotide sequence similarity varied from 74 and 78% to 100% for 5'-PCR and NS5-PCR fragments, respectively. Additionally, five overlapping PCR fragments comprising an approximately 2.0-kb structural region of the HGV genome were sequenced from each of five sera obtained from three United States residents. These sequences were compared to 20 published sequences comprising the same region of the HGV genome. Nucleotide and deduced amino acid sequences obtained from different individuals were homologous from 82.9 to 93. 6% and from 90.4 to 99.0%, respectively. Sequences obtained from follow-up specimens were almost identical. Comparative analysis of deduced amino acid sequences of the HGV structural proteins and hepatitis C virus (HCV) structural proteins combined with an analysis of predicted secondary structures and hydrophobic profiles allowed prediction of processing sites within the HGV structural proteins. A phylogenetic sequence analysis performed on the 2.0-kb structural region supports the existence of three previously identified HGV genetic groups. However, phylogenetic analysis performed on only small DNA fragments yielded inconsistent genetic grouping and failed to confirm the existence of genetic groups. Thus, in contrast to HCV where almost any region can be used for genotyping, only large or carefully selected genome fragments can be used to identify consistent HGV genetic groups.  (+info)

RNA-Stimulated ATPase and RNA helicase activities and RNA binding domain of hepatitis G virus nonstructural protein 3. (4/233)

Hepatitis G virus (HGV) nonstructural protein 3 (NS3) contains amino acid sequence motifs typical of ATPase and RNA helicase proteins. In order to examine the RNA helicase activity of the HGV NS3 protein, the NS3 region (amino acids 904 to 1580) was fused with maltose-binding protein (MBP), and the fusion protein was expressed in Escherichia coli and purified with amylose resin and anion-exchange chromatography. The purified MBP-HGV/NS3 protein possessed RNA-stimulated ATPase and RNA helicase activities. Characterization of the ATPase and RNA helicase activities of MBP-HGV/NS3 showed that the optimal reaction conditions were similar to those of other Flaviviridae viral NS3 proteins. However, the kinetic analysis of NTPase activity showed that the MBP-HGV/NS3 protein had several unique properties compared to the other Flaviviridae NS3 proteins. The HGV NS3 helicase unwinds RNA-RNA duplexes in a 3'-to-5' direction and can unwind RNA-DNA heteroduplexes and DNA-DNA duplexes as well. In a gel retardation assay, the MBP-HGV/NS3 helicase bound to RNA, RNA/DNA, and DNA duplexes with 5' and 3' overhangs but not to blunt-ended RNA duplexes. We also found that the conserved motif VI was important for RNA binding. Further deletion mapping showed that the RNA binding domain was located between residues 1383 and 1395, QRRGRTGRGRSGR. Our data showed that the MBP-HCV/NS3 protein also contains the RNA binding domain in the similar domain.  (+info)

Identification of a novel genotype of hepatitis G virus in Southeast Asia. (5/233)

Hepatitis G virus (HGV) isolates obtained from 20 Myanmarese and 10 Vietnamese subjects were analyzed. A cluster of isolates not belonging to any known genotype of HGV was found in five Myanmarese subjects and three Vietnamese subjects by phylogenetic analysis, and we classified this new genotype as type 4. These results revealed that the HGV genome can be classified into at least four major genotypes.  (+info)

Seroprevalence of hepatitis B virus, hepatitis C virus and GB virus-C infections in Siberia. (6/233)

We studied the seroprevalence of hepatitis B virus (HBV), hepatitis C virus (HCV) and GB virus-C (GBV-C) infections in 348 Siberian natives who lived in the Kamchatka Peninsula of Russia. Of 348 samples studied, the seroprevalence of HBsAg and anti-HBs were 11.8% (41 of 348 samples) and 35.9% (125 of 348 samples), respectively. The prevalence of HCV infection was 1.4% (5 of 348 samples), and that of GBV-C RNA, using RT-PCR methods, was 7.5% (26 of 348 samples). In Siberia, the prevalences of HBV and GBV-C infections were about tenfold higher than those in Japan. The prevalence of HBsAg in subjects under 50 years of age was significantly higher than that in those over 50 years old (P < 0.05). Because HBV infection is highly endemic in Siberia, we propose that the community-based mass immunization must be conducted as soon as possible in this area.  (+info)

Analysis of hepatitis G virus (HGV) RNA, antibody to HGV envelope protein, and risk factors for blood donors coinfected with HGV and hepatitis C virus. (7/233)

Serologic, biochemical, and molecular analyses were used to study hepatitis G virus (HGV), antibody to the HGV envelope protein (anti-E2), risk factors, clinical significance, and the impact of HGV on coexistent hepatitis C virus (HCV). Among 329 donors with confirmed HCV infection, 12% were HGV RNA-positive and 44% were anti-E2-positive (total exposure, 56%). HGV RNA and anti-E2 were mutually exclusive except in 9 donors (1.5%); 8 of 9 subsequently lost HGV RNA but anti-E2 persisted. HGV had little impact on alanine aminotransferase, aspartate aminotransferase, or gamma-glutamyl transpeptidase in donors with HGV infection alone or those coinfected with HCV. A multivariate analysis showed that intravenous drug abuse was the leading risk factor for HGV transmission, followed by blood transfusion, snorting cocaine, imprisonment, and a history of sexually transmitted diseases. In summary, HGV and HCV infections were frequently associated and shared common parenteral risk factors; HGV did not appear to cause hepatitis or to worsen the course of coexistent hepatitis C.  (+info)

Early acquisition of TT virus (TTV) in an area endemic for TTV infection. (8/233)

TT virus (TTV) is widely distributed, with high frequencies of viremia in South America, Central Africa, and Papua New Guinea. The incidence and timing of infection in children born in a rural area of the Democratic Republic of Congo was investigated. TTV viremia was detected in 61 (58%) of 105 women attending an antenatal clinic and in 36 (54%) of 68 infants. Most infants acquired the infection at >/=3 months postpartum. Surprisingly, TTV infection was detected in a large proportion of children with TTV-negative mothers (13 [43%] of 30). Nucleotide sequences of TTV-infected children were frequently epidemiologically unlinked to variants detected in the mother. These three aspects contrast with the maternal transmission of hepatitis G virus/GB virus C in this cohort and suggest an environmental source of TTV infection comparable to hepatitis A virus and other enterically transmitted infections.  (+info)

Background & Aims : GB virus-C/hepatitis G virus GBV-C/HGV is a newly identified flavivirus, and little is known about its clinical significance. GBV-C/HGV was investigated in different populations, and its coinfection was investigated in patients with liver disease in Taiwan where hepatitis B and C are endemic. Methods : Viral RNA was studied...
Other articles where Hepatitis G virus is discussed: hepatitis: Hepatitis F and G: …virus isolated in 1996, the hepatitis G virus (HGV), is believed to be responsible for a large number of sexually transmitted and bloodborne cases of hepatitis. HGV causes acute and chronic forms of the disease and often infects persons already infected with HCV.
Hepatitis G virus (HGV) is an RNA virus belongs to the Flaviviridae family. It is mainly transmitted through exposure to contaminated blood although other routes may also exist. Patients with chronic renal failure are at high risk of acquiring HGV because they require frequent blood transfusions. HGV infection can be diagnosed by demonstrating viremia in patient sample by reverse transcriptase (RT) PCR. The coinfection of hepatitis G with hepatitis В, С or D is detected frequently.. ...
Patients who only had HCV infection did not differ from patients with HCV and HGV co-infection in clinical presentation, HCV RNA level, or response of HCV to interferon-alpha therapy. Thus, HGV infection had no apparent influence on the clinical or virologic course of HCV infection. Hepatitis G viru …
Hepatitis G virus/ GB virus C is a novel flavivirus recently detected in hepatitis non A-E cases. In this study, the presence of this virus in chronic non-B, non-C hepatitis patients was evaluated using GBV-C specific PCR and this virus was detected in one out of thirteen patients. This patient has presented a severe liver failure, has lived for a long time in the Western Amazon basin and no other cause for this clinical picture was reported. The impact of the discovery of this new agent is still under evaluation throughout the world. The study of the prevalence of this virus among chronic hepatitis patients and healthy individuals (as blood donors) will furnish subside to evaluate its real pathogenicity ...
We have determined the nucleotide sequence of the 5-terminal region of the hepatitis G virus (HGV) genome in 11 hepatitis patients from three cities in China. Phylogenetic analyses revealed that the Chinese isolates were genetically distinct from previously described West African isolates (type 1) and American, European and East African isolates (type 2), with a mean sequence divergence of approximately 10%. The mean divergence between isolates from Lanzhou, in the northwest of China, and those from Shanghai and Nanjing, on the east coast of China, was 5% (range 3-7%). The isolates from Shanghai and Nanjing were closely related to a common strain in Japan, while some of those from Lanzhou were closely related to a southeast Asian type 3 isolate. Thus, the Chinese isolates belong to the type 3 variant of HGV.
Evidence indicates that the GBV-C or hepatitis G virus can cause persistent infection in humans, but little is known on the importance of vertical transmission. To assess the risk of mother-to-infant transmission and the clinical outcome of infected babies, we investigated 175 anti-HCV positive mothers and followed-up their children for 3-33 months. GBV-C RNA was detected by RT-PCR and anti-E2 antibody was assayed by EIA. Thirty-four (19.4%) women were GBV-C RNA positive and transmission occurred to 21 (61.8%) babies; 20 (95.2%) acquired GBV-C alone, and one (4.8%) GBV-C and HCV. Maternal factors such as intravenous drug use, HIV coinfection, HCV-RNA positivity, and type of feeding were not correlated with GBV-C transmission. GBV-C RNA remained persistently positive in all infected babies but one baby who seroconverted to anti-E2. Seven (35%) babies with GBV-C alone developed marginally elevated ALT; the baby with HCV and GBV-C co-infection had the highest ALT peak value (664 IU/l). Seven of the ...
Adams, N. J., Prescott, L. E., Jarvis, L. M., Lewis, J. C., McClure, M. O., Smith, D. B. & Simmonds, P. (1998). Detection in chimpanzees of a novel flavivirus related to GB virus-C/hepatitis G virus. J Gen Virol 79 ( Pt 8), 1871-1877. [PMID 9714234]. Agnello, V., Abel, G., Elfahal, M., Knight, G. B. & Zhang, Q. X. (1999). Hepatitis C virus and other Flaviviridae viruses enter cells via low density lipoprotein receptor. Proc Natl Acad Sci U S A 96, 12766-12771. [PMID 10535997]. Baechlein, C., Fischer, N., Grundhoff, A., Alawi, M., Indenbirken, D., Postel, A., Baron, A. L., Offinger, J., Becker, K., Beineke, A., Rehage, J. & Becher, P. (2015). Identification of a Novel Hepacivirus in Domestic Cattle from Germany. J Virol 89, 7007-7015. [PMID 25926652]. Bukh, J., Kim, J. P., Govindarajan, S., Apgar, C. L., Foung, S. K., Wages, J., Jr., Yun, A. J., Shapiro, M., Emerson, S. U. & Purcell, R. H. (1998). Experimental infection of chimpanzees with hepatitis G virus and genetic analysis of the virus. J ...
Virus-host biological interaction is a continuous coevolutionary process involving both host immune system and viral escape mechanisms. Flaviviridae family is composed of fast evolving RNA viruses that infects vertebrate (mammals and birds) and/or invertebrate (ticks and mosquitoes) organisms. These host groups are very distinct life forms separated by a long evolutionary time, so lineage-specific anti-viral mechanisms are likely to have evolved. Flaviviridae viruses which infect a single host lineage would be subjected to specific host-induced pressures and, therefore, selected by them. In this work we compare the genomic evolutionary patterns of Flaviviridae viruses and their hosts in an attempt to uncover coevolutionary processes inducing common features in such disparate groups. Especially, we have analyzed dinucleotide and codon usage patterns in the coding regions of vertebrate and invertebrate organisms as well as in Flaviviridae viruses which specifically infect one or both host types. ...
Abbas, Y. M., Pichlmair, A., Gorna, M. W., Superti-Furga, G. & Nagar, B. (2013). Structural basis for viral 5-PPP-RNA recognition by human IFIT proteins. Nature 494, 60-64. [PubMed]. Adams, N. J., Prescott, L. E., Jarvis, L. M., Lewis, J. C., McClure, M. O., Smith, D. B. & Simmonds, P. (1998). Detection in chimpanzees of a novel flavivirus related to GB virus-C/hepatitis G virus. J Gen Virol 79 ( Pt 8), 1871-1877. [PubMed]. Baechlein, C., Fischer, N., Grundhoff, A., Alawi, M., Indenbirken, D., Postel, A., Baron, A. L., Offinger, J., Becker, K., Beineke, A., Rehage, J. & Becher, P. (2015). Identification of a Novel Hepacivirus in Domestic Cattle from Germany. J Virol 89, 7007-7015. [PubMed]. Baechlein, C., Grundhoff, A., Fischer, N., Alawi, M., Hoeltig, D., Waldmann, K. H. & Becher, P. (2016). Pegivirus Infection in Domestic Pigs, Germany. Emerging Infect Dis 22, 1312-1314. [PubMed]. Bailey, A. L., Lauck, M., Ghai, R. R., Nelson, C. W., Heimbruch, K., Hughes, A. L., Goldberg, T. L., Kuhn, J. H., ...
Proposals are described for the assignment of recently reported viruses, infecting rodents, bats and other mammalian species, to new species within the Hepacivirus and Pegivirus genera (family Flaviviridae). Assignments into 14 Hepacivirus species (Hepacivirus A- N) and 11 Pegivirus species (Pegivirus A- K) are based on phylogenetic relationships and sequence distances between conserved regions extracted from complete coding sequences for members of each proposed taxon. We propose that the species H epatitis C virus is renamed Hepacivirus C in order to acknowledge its unique historical position and so as to minimize confusion. Despite the newly documented genetic diversity of hepaciviruses and pegiviruses, members of these genera remain phylogenetically distinct, and differ in hepatotropism and the possession of a basic core protein; pegiviruses in general lack these features. However, other characteristics that were originally used to support their division into separate genera are no longer definitive
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臺大位居世界頂尖大學之列,為永久珍藏及向國際展現本校豐碩的研究成果及學術能量,圖書館整合機構典藏(NTUR)與學術庫(AH)不同功能平台,成為臺大學術典藏NTU scholars。期能整合研究能量、促進交流合作、保存學術產出、推廣研究成果。. To permanently archive and promote researcher profiles and scholarly works, Library integrates the services of NTU Repository with Academic Hub to form NTU Scholars.. ...
What can I take for chronic muscle pain? 75 yr. Old. Hep C for 20-30 yr. Family Doctor tells me to take Tylenol..dont take it as condition is chronic & severe. Cant sleep due to it.
Just curious about the possibility of getting Hep C from my partner while having sex, oral, or just kissing? What are the risks, and what precautions if any should we be taking. Is abstinence the answe...
Disclosed are compounds, compositions and methods for treating viral infections caused by a flaviviridae family virus, such as hepatitis C virus. Such compounds are represented by Formula I as follows: ##STR00001## wherein, Y, W, W.sup.1 and W.sup.2 are as defined herein.
in Acta Gastro-Enterologica Belgica (1999), 62(1), 13-15. Background : Recently, a novel blood-borne virus has been identified and named hepatitis G virus. Transfusion is the main route of transmission. It is known that patients on maintenance dialysis are more ... [more ▼]. Background : Recently, a novel blood-borne virus has been identified and named hepatitis G virus. Transfusion is the main route of transmission. It is known that patients on maintenance dialysis are more susceptible to infections with parenterally-transmitted viruses than the general population. The aim of the present study was to determine the prevalence of hepatitis G infection in a Belgian dialysis unit. Methods: The entire population of our dialysis unit (82 patients) was tested for the presence of hepatitis G virus (HGV) by reverse transcriptase polymerase chain reaction. History of transfusion or renal transplantation coinfections with hepatitis B and C viruses, and serum aminotransferase levels were also tested. ...
Hepatitis G is a newly discovered form of liver inflammation caused by hepatitis G virus (HGV), believed to be a distant relative of the hepatitis C virus. HGV, also called hepatitis GB virus, was first described early in 1996. Little is known about the frequency of HGV infection, the nature of the illness, or how to prevent it. What is known is that transfused blood containing HGV has caused some cases of hepatitis. For this reason, patients who require large amounts of blood or blood products are at risk of hepatitis G. Often patients with hepatitis G are infected at the same time by the hepatitis B or C virus, or both. In about three of every thousand patients with acute viral hepatitis, HGV is the only virus present. There is some indication that patients with hepatitis G may continue to carry the virus in their blood for many years, and so might be a source of infection in others ...
Blood-borne hepatitis is a well-known complication in patients with bleeding disorders. A recently discovered parentally transmitted virus, hepatitis G [GB virus C (GBV-C)] has an increased prevalence in patients with haemophilia. Clotting factor concentrates derived from pools of human plasma currently undergo viral inactivation techniques known to be effective against hepatitis B, C and HIV; however, the effectiveness of current purification and viral inactivation techniques against newly discovered viruses such as GBV-C is unknown. A total of 37 vials of clotting factor concentrates manufactured in the USA from 1981 to 1995 were tested for the presence of GBV-C virus. All samples that did not undergo a specific viral inactivation step were positive for GBV-C. Viral inactivation techniques that did not uniformly remove GBV-C included vapour heat treatment and dry heat treatments for less than 144 h. All samples treated by pasteurization, solvent detergent or dry heat for 144 h, were negative for the
One year later, after a reform of the education programme, another group of first-year students were tested what is tadalafil used for (212 girls and 117 boys) in the same schools. We determined by sonography the prevoiding bladder volume needed to void a sufficient amount of urine.. Evaluation of the identification power of RPLC analyses in the screening for drug compounds. A second post-processing requirement is the frequency axis assignment. Detection of tadalafila 5mg hepatitis G virus RNA in persons with and without known risk factors for blood-borne viral infections in Sweden and Honduras. However, some events appeared to be predictors of patient admission rates. To explore the feasibility of using social media-based patient networks to gather qualitative data on patient-reported outcome (PRO) concepts tadalafil dosage relevant to chronic lymphocytic leukemia (CLL). Implementation of pattern-mixture models in randomized clinical trials. An estrogen receptor mutant tadalafil kopen nederland ...
Discoveries of new human viruses and new technologies for their detection have made, and will continue to make, major contributions to the safety of blood transfusion. This article discusses the practical issues involved in the implementation of additional serological screening tests for viruses such as human T-lymphotropic virus, and reviews current information on the prevalence and pathogenicity of more recently discovered viruses, such as hepatitis G virus (HGV) or GB virus-C (GBV-C) and human herpes virus 8, a potential aetiological agent of Kaposis sarcoma. Progress in the technology behind nucleic acid amplification techniques, such as the polymerase chain reaction (PCR), makes direct detection of viruses such as human immunodeficiency virus and hepatitis C virus possible. The use of such methods for screening will allow the earlier detection of acutely-infected individuals and the elimination of transmission from window period donations before seroconversion for antibody. Establishing a
Infections with hepatitis B and C viruses (HBV, HCV) are widespread in human populations throughout the world, and are major causes of chronic liver disease and liver cancer. HBV, HCV and the related hepatitis G virus or GB virus C (referred to here as HGV/GBV-C) are capable of establishing persistent, frequently lifelong infections characterized by high levels of continuous replication. All three viruses show substantial genetic heterogeneity, which has allowed each to be classified into a number of distinct genotypes that have different geographical distributions and associations with different risk groups for infection. Information on their past transmission and epidemiology might be obtained by estimation of the time of divergence of the different genotypes of HCV, HBV and HGV/GBV-C using knowledge of their rates of sequence change. While information on the latter is limited to short observation periods and is therefore subject to considerable error and uncertainty, the relatively recent times of
Background: GB virus C (GBV-C) or hepatitis G virus (HGV) is a newly discovered and enveloped RNA positive-stranded flavivirus-like particle which has not yet been proven to have major negative effects on liver. Army hospitals in Tehran were included. Serum HIV antibody (Ab) HCV antibody and HBS antigen (Ag) were assessed. Demographic data such as gender age blood group cause of renal failure dialysis onset and duration were collected from medical files. GBV-C/HGV was evaluated by nested reverse transcription polymerase chain reaction (RT-PCR) method. Then all data were analyzed by SPSS ver. 13. Etomoxir Results: In total 81 males and 57 females were included. The mean age of patients was 62.16 ± 14.86 years. Six (4.3%) had positive results for GBV-C/HGV by RT-PCR. Except gender (P = 0.045) and duration of dialysis in a week (P < 0.001) other demographic factors revealed no significant difference (P > 0.05). All patients had negative results for HIV Ab HCV Ab and HBS Ag. Conclusions: Overall ...
Results show non-pathogenic E1 GB virus C protein as an attractive source of peptides for development of novel anti-HIV therapies- Global Medical Discovery
Results: Combined GBV-C RNA and anti-E2 prevalances ranged from 6.5% in children, 13.3% in blood donors, 14% in pregnant women, 22.5% in haemodialysis patients, 80% in anti-HCV positive donors and 88.6% in IVDUs and 85.7% in adult haemophiliacs. Children had the lowest antibody rate, 1.1%, compared with 10.8% for blood donors rising to 45.7% for IVDUs and to 74.6% for haemophiliacs. In contrast current infection rates were comparable for children, blood donors and pregnant women at 5.4%, 2.6% and 6%. These rates rose to 11.1% for haemophiliacs, 24.3% for anti-HCV donors and 48.6% for IVDUs. Ten of 12 donors had persistent vireamia while two had recent infections, one with apparent resolution. 47% of donors who had lived in the Pacific Islands had GBV-C exposure compared to 16% who had not lived in the Pacific. (P,0.01). Spending evening outdoors was also associated with HGB exposure ...
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Ive decided to start posting abstracts and links to papers that I find interesting. Todays topic: GB virus C.. GBV-C is a non-pathogenic virus that infects humans. This 2014 paper details the impact of co-infection with HIV-1. Ultimately they conclude that GBV-C co-infection may be beneficial to HIV-1 patients. However, the benefits of GBV-C are not apparent when patients are treated with anti-retroviral therapy.. Impact of GB virus C viraemia on clinical outcome in HIV-1-infected patients: a 20-year follow-up study.. D Ernst, M Greer, R Akmatova, S Pischke, H Wedemeyer, H Heiken, HL Tillmann, RE Schmidt and M Stoll. HIV Medicine v. 15 p. 245-250. ...
The prevalence of anti-HCV among patients on hemodialysis is consistently higher than in the general population, indicating that patients on hemodialysis programs are at risk of acquiring HCV infection. The response to interferon alpha 26 (IFN -alpha 26) therapy in chronic C hepatitis depends on viral and host factors. We treated 22 chronic C hepatitis uremic patients with IFN -alpha 26 (3 MU three times a week) and compared interferon responsive and unresponsive patients with regard to HLA II genes. HLA II genes were investigated by PCR-SSP low resolution, anti-HCV with ELISA II and HCV-RNA with reverse transcriptase nested PCR. Findings: HLA DRB1*13 is 50% positive in the non-responder group (four women, four men, mean age; 28.8 +/- 11.9 years) and 7% in the responder group (five women, nine men, mean age; 32.2 +/- 7.8 years) ( ...
Remedy Health Media: About Remedy Health Media , Contact Remedy Health Media , Terms of Use , Privacy Policy. TheBodyPRO.com is a service of Remedy Health Media, LLC, 750 3rd Avenue, 6th Floor, New York, NY 10017. TheBodyPRO.com and its logos are trademarks of Remedy Health Media, LLC, and its subsidiaries, which owns the copyright of TheBodyPRO.coms homepage, topic pages, page designs and HTML code. General Disclaimer: TheBodyPRO.com is designed for educational purposes only and is not engaged in rendering medical advice or professional services. The information provided through TheBodyPRO.com should not be used for diagnosing or treating a health problem or a disease. It is not a substitute for professional care. If you have or suspect you may have a health problem, consult your health care provider.. ...
Non-primate hepacivirus (NPHV), equine pegivirus (EPgV) and Theilers disease associated virus (TDAV) are newly discovered members of two genera in the Flaviviridae family, Hepacivirus and Pegivirus respectively, that include human hepatitis C virus (HCV) and human pegivirus (HPgV). To investigate their epidemiology, persistence and clinical features of infection, large cohorts of horses and other mammalian species were screened for NPHV, EPgV and TDAV viraemia and for past exposure through serological assays for NPHV and EPgV-specific antibodies. NPHV antibodies were detected in 43% of 328 horses screened for antibodies to NS3 and core antibodies, of which three were viraemic by PCR. All five horses that were stablemates of a viraemic horse were seropositive, as was a dog on the same farm. With this single exception, all other species were negative for NPHV antibodies and viraemia: donkeys (n=100), dogs (n=112), cats (n=131), non-human primates (n=164) and humans (n=362). EPgV antibodies to NS3 were
Medical definition of Flaviviridae: a family of single-stranded RNA viruses that have a spherical virion with a capsid composed of a single protein…
Study Flashcards On M2-C3-L93 --| Flaviviridae, Togaviridae, Bunyaviridae at Cram.com. Quickly memorize the terms, phrases and much more. Cram.com makes it easy to get the grade you want!
PubMed comprises more than 30 million citations for biomedical literature from MEDLINE, life science journals, and online books. Citations may include links to full-text content from PubMed Central and publisher web sites.
Odolaren bidez birusa gibelera iristen da eta bertan hepatozitoak kaltetzen ditu. Infektatuen gehiengoak hasieran ez du inongo sintomarik. Birusaren eramaileak dira eta odolaren bidez beste pertsona batzuk kutsa ditzakete, baina asintomatikoak daudenez ez dira arrisku horretaz ohartzen. Infektatutako gizabanako gutxi batzuek nekea, goragaleak, pisu-galera eta ikteriziarik gabeko hepatitisaren ohiko sintomak ere dituzte [4] baina -esan bezala- gehienek ez dute inongo sintomarik garatzen. Azken hauengan gaitza antzematen da odol-analisi arrunt batean (transaminasak eta beste entzima hepatikoen balioak aztertuz). Gaixo gutxi batzuen immunitate-sistemak birusa deuseztatzen du, baina infektatuen %80ak hepatitis kronikoa garatuko du. Birusak urte askotan gibela kaltetzen du, eta epe aldakor batean (10-30 urte) zirrosia eta gibeleko minbizia eragin dezake. Gibela oso kaltetuta dagoenean, sintomak nabariagoak dira: nekea, sukarra, goragaleak, ikterizia, sabelmina.... Gaitzaren progresio-erritmoa adina, ...
|strong|Rabbit anti West Nile Virus antibody|/strong| recognizes the ~14 kDa core protein of West Nile Virus (WNV). WNV belongs to the Flaviviridae family which includes closely viruses such as D…
Chang K.-C; Chang M.-H; Lee C.-N; Chang C.-H; Wu J.-F; Ni Y.-H; Wen W.-H; MING-KWANG SHYU ; Lai M.-W; Chen S.-M; Hu J.-J; Lin H.H; Hsu J.-J; Mu S.-C; Lin Y.-C; Liu C.-J; Chen D.-S; Lin L.-H; Chen H.-L; Hwa H.-L; Su Y.-N; Shih J.-C; Chao K.-H; Hsu H.-Y; Chiu Y.-C; Su T.-H; Lin C.-C; Lin P.-Y; Yang W.-R; Zhao L.-L; Yang C.-K; Chang Y.-K; Chen K.-H; Lin Y.-H; Chen H.-J; Pan H.-S; Lau B.-H; Lee C.-L; Cheng P.-J; Chang Y.-L; Chiueh H.-Y; Wang T.-H; Lo L.-M; Hsieh C.-L; Cheng S.-W; Tsai M.-S; She B.-Q; Koh K.-J; Hung Y.-L; Lin H.-M; Peng F.-S; Wu T.-C; Chen C.-Y; Chen C.-P; Huang J.-P; Yeung C.-Y; Lin C.-J; Chiu W.-T; Wang D.-S; Lin W.-T; Hwang K.-S; Huang C.-F; The Taiwan Study Group for the Prevention of Mother-to-Infant Transmission of HBV (PreMIT study) ...
A brand new and ever more powerful strain of the Virus was released in 2002 to replace the entire B Series line-up. First up is the flag-ship desktop module, the Virus C. Polyphony is expanded from 24 to 32 voices! Otherwise the synth engine is virtually the same as the B series. Three oscillators, one sub osc., two independent multi-mode filters, two ADSTR envelopes, three LFOs, 16 arpeggiators, 32-band vocoder, five FM synthesis modes and 16 part multitimbrality. The C series offers an expanded mod matrix with 6 source and 9 destination slots, 98 effects, wood side panels, and more! A new 3-band EQ section is available as well as an Undo/Redo function. The operating systems can be easily upgraded via MIDI and there is a free SoundDiver Virus editor/librarian for Windows and MacOS systems available. It has been used by Nine Inch Nails, KMFDM, and Max Graham.. ...
357 Guillen-Nunez, E Deadlines Final Book WAIT LIST HLTH 91C Hepatitis ABCs 1.0 72848 001 Lec T 07:10-09:00PM 10/21-12/09 MUB ...
The purpose of this study is to examine the effect of GB virus C (GBV-C) on the natural history of chronic hepatitis C virus (HCV) infection in subjects co-infected with HIV and HCV. The other aspect of the study is to assess the effect of GBV-C on the severity of liver disease due to chronic hepatitis C in subjects co-infected with HIV and HCV. This will be done by determining the point prevalence of co-infection retrospectively then following that cohort prospectively. In addition, further individuals will be recruited in a prospective manner ...
The purpose of this study is to examine the effect of GB virus C (GBV-C) on the natural history of chronic hepatitis C virus (HCV) infection in subjects co-infected with HIV and HCV. The other aspect of the study is to assess the effect of GBV-C on the severity of liver disease due to chronic hepatitis C in subjects co-infected with HIV and HCV. This will be done by determining the point prevalence of co-infection retrospectively then following that cohort prospectively. In addition, further individuals will be recruited in a prospective manner ...
GB virus C (GBV-C), which is highly prevalent among HIV/AIDS, seemed to slow the HIV disease progression. The HIV/GBV-C co-infected individuals may represent an interesting model for the investigation of the role played by HIV infection and/or the immune system in driving the evolution of the GBV-C viral populations. The present study investigated the prevalence and population dynamics of GB virus C in HIV infected individuals representing 13 geographic regions of Hubei Province of China. Approximately 37% of HIV-1 infected individuals were infected with GBV-C and genotype 3 is appeared to be predominant. Utilizing the 196 complete E2 nucleotide sequence data from 10 HIV/GBV-C infected individuals and employing coalescence based phylogenetic approaches; the present study has investigated the intra-host dynamics of GBV-C. The results revealed patient-specific unique GBV-C viral lineages and each viral lineage showed the evidence of rapid population expansion in respective HIV-1 infected patients, thus
The sexual partners of men with hepatitis C virus (HCV) infection have a relatively low prevalence of the disease, yet cases of sexual transmission have been reported. With the discovery of a new HCV-related virus named hepatitis G (HGV) or GBV-C, the issue of sexual transmission has become more complicated. It is now recognized that HGV and GBV-C are two distinct isolates of the same virus, to be labeled HGV/GBV-C. Previous studies indicate that HGV/GBV-C infection may occur in up to 7 percent of the population and may infect up to 30 percent of persons at risk for bloodborne infections, such as intravenous drug users. Semprini and colleagues examined semen and serum samples from men infected with hepatitis C to determine whether HCV RNA and HGV/GBV-C RNA are present in semen.. The 90 men in the study were former intravenous drug users. All tested positive for anti-HCV antibodies. Of the group, 27 were also positive for human immunodeficiency virus (HIV) infection, and 10 were positive for ...
GB virus type C (GBV-C) is a common human flavivirus that has been associated with prolonged survival in HIV-positive individuals in several, though not all, epidemiological studies. There are five distinct GBV-C genotypes that are geographically localized, and it has been speculated that GBV-C genotypic differences may explain variable outcomes observed in different clinical studies. Expression of an 85 aa fragment of the GBV-C NS5A phosphoprotein (genotype 2) in a CD4+ T cell line (Jurkat) resulted in inhibition of HIV replication, mediated in part by decreased surface expression of the HIV coreceptor CXCR4 and upregulation of SDF-1. We expressed the NS5A protein from genotypes 1, 2, 3 and 5 in Jurkat cells, and demonstrated that all genotypes inhibited HIV replication. Further deletion mapping demonstrated that expression of a 30 aa fragment resulted in decreased CXCR4 surface expression, upregulation of SDF-1 and inhibition of HIV replication.
GB virus C (GBC) and HIV Co-infection: Researchers at the University of Iowa used ARRA funding to expand their ongoing research to better understand the apparent protective effect of GB virus C (GBV-C) infection on the progression of HIV in people coinfected with the two viruses. Co-infection with GBV-C, a virus that is not known to cause any disease, is common in HIV-infected individuals. The researchers found that GBV-C appears to inhibit specific immune signaling pathways and immune activation, which is a key process in HIV disease progression. In addition, the presence of GBV-C in the bloodstream was found to be associated with a reduction in programmed cell death of activated T-cells (a type of immune cell). These GBV-C-associated T-cell effects could contribute to the observed protective effect of GBV-C co-infection in HIV-infected individuals.4 ...
Journal of Shahid Sadoughi University of Medical Sciences ماهنامه علمی پژوهشی دانشگاه علوم پزشکی و خدمات بهداشتی درمانی شهید صدوقی یزد
The Zika virus is a single stranded RNA virus of the Flaviviridae family transmitted primary by Aedes mosquitoes, although other modes of transmission are possible including mother-to-child, sexual and blood transfusions. The virus is usually self-limiting with symptoms similar to dengue and chikungunya, and work is on-going to establish a link between the virus and […]. [Read More] ...
Yellow fever is a mosquito borne disease caused by a virus from the Flaviviridae family, called the yellow fever virus. This virus is transmitted via female
Louping-ill Louping-ill is an acute viral disease of the central nervous system transmitted by sheep ticks (Ixodes ricinus). The disease can also spread via droplet infection. The disease is caused by a member of the Flaviviridae family. It is antigenically closely related (i.e.
The latest issue of the Annals of Internal Medicine investigates reducing the risk for mother-to-infant transmission of Hepatitis C virus.. ...
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Pregnancy in HIV-infected women. Counseling and care--12 years experiences and results.: Although it is possible to reduce mother-to-infant transmission by zid
Definition: Zika virus is a member of the virus family Flaviviridae and the genus Flavivirus, transmitted by daytime-active Aedes mosquitoes, such as A. aegypti
The virus family Flaviviridae encompasses several viruses, including (re)emerging viruses which cause widespread morbidity and mortality throughout the world. Members of this virus family are positive-strand RNA viruses and replicate their genome in close association with reorganized intracellular host cell membrane compartments. ...
No Evidence of Mosquito Involvement in the Transmission of Equine Hepacivirus (Flaviviridae) in an Epidemiological Survey of ...
Hepacivirus genus (family Flaviviridae) is an expressive example of a viral genus that grew with the advent of metagenomics. ... Hepacivirus genus (family Flaviviridae) is an expressive example of a viral genus that grew with the advent of metagenomics. ... O gênero Hepacivirus (família Flaviviridae) é um exemplo expressivo de gênero viral que cresceu rapidamente com o advento da ... O gênero Hepacivirus (família Flaviviridae) é um exemplo expressivo de gênero viral que cr ... ...
Flaviviridae, Flavivirus) in Blackbirds (Turdus merula) and Mosquitoes (Culex pipiens, Cx. modestus) in the Czech Republic ( ...
Arenaviridae, Bunyaviridae, Filoviridae and Flaviviridae are all RNA viruses that can induce a coagulopathy in infected ... Platelets; Arenaviridae; Bunyaviridae; Filoviridae; Flaviviridae; Dengue; Viral hemorrhagic fever; Disseminated intravascular ...
Viruses; Flaviviridae. Comments. None. Copyright PROSITE is copyrighted by the SIB Swiss Institute of Bioinformatics and ...
Flaviviridae. 08/05/2021. 11:30 - 12:30. https://eu.bbcollab.com/guest/ac6ae77a4822468fa268b4bdb8aeb629 ...
Lim, S. P., Sonntag, L. S., Noble, C., Nilar, S. H., Ng, R. H., Zou, G., Monaghan, P., Chung, K. Y., Dong, H., Liu, B., Bodenreider, C., Lee, G., Ding, M., Chan, W. L., Wang, G., Jian, Y. L., Chao, A. T., Lescar, J., Yin, Z., Vedananda, T. R. & 2 others, Keller, T. H. & Shi, P. Y., Feb 25 2011, In: Journal of Biological Chemistry. 286, 8, p. 6233-6240 8 p.. Research output: Contribution to journal › Article › peer-review ...
The focus of Protinhi Therapeutics is on upcoming viral threats, including flaviviridae (e.g., dengue), chikungunya and ...
Usutu virus (USUV), a member of the mosquito-borne clade within the Flaviviridae family, was responsible for avian mortality in ... abstract = "Usutu virus (USUV), a member of the mosquito-borne clade within the Flaviviridae family, was responsible for avian ... N2 - Usutu virus (USUV), a member of the mosquito-borne clade within the Flaviviridae family, was responsible for avian ... AB - Usutu virus (USUV), a member of the mosquito-borne clade within the Flaviviridae family, was responsible for avian ...
... required for the effective inactivation of zoonotic viruses belonging to the medically important families of Flaviviridae, ... required for the effective inactivation of zoonotic viruses belonging to the medically important families of Flaviviridae, ... required for the effective inactivation of zoonotic viruses belonging to the medically important families of Flaviviridae, ... required for the effective inactivation of zoonotic viruses belonging to the medically important families of Flaviviridae, ...
13:00 - 13:30 , Keynote Lecture 6: Mitochondrial manipulations by Flaviviridae. Laurent Chatel-Chaix - National Institute of ...
They all belong to the Flaviviridae family. Research from the NIHs Biomedical Research Centre suggests that, due to ...
2. Identification of novel host factors in Flaviviridae infections 3. Dissecting the architecture of nuclear matrix fibers. ...
FAO Animal hog cholera, which is caused by a Pestivirus in the Flaviviridae family. The PDF file you selected should load here ...
Zika Virus infection is caused by the Zika Virus (ZIKV) belonging to the Flaviviridae family. The virus is primarily ...
Zika virus (ZIKV) is a member of the Flaviviridae virus family and the Flavivirus genus. In humans, it initially causes a mild ...
Dengue virus is a mosquito-borne, single positive-stranded RNA virus of the family Flaviviridae; genus Flavivirus. ...
Chapter 54 - Alphaviruses (Togaviridae) and Flaviviruses (Flaviviridae). Pärit leheküljelt "https://et.wikipedia.org/w/index. ...
It is a mosquito-borne single positive-stranded RNA virus of the family Flaviviridae and genus Flavivirus. Science illustration ... It is a mosquito-borne single positive-stranded RNA virus of the family Flaviviridae and genus Flavivirus. ...
A digitally-colorized transmission electron micrograph of Zika virus, which is a member of the family Flaviviridae. Virus ...
... infectious disease that affects humans and animals which is caused by the dengue virus belonging to the Flaviviridae family and ...
West Nile virus is a Flavivirus in the Flaviviridae family, similar to St Louis encephalitis virus, Dengue and Zika. ...
This is because both these viruses belong to the same family called the Flaviviridae and the same mosquitoes transmit them. ...
Aelod o deulur Flaviviridae ydywr firws Zika ar genws Flavivirus, syn cael ei gario liw dydd gan y mosgito Aedes aegypti. Dawr ...
2019;10:753 Abstract Zika virus (ZIKV) is an arthropod-borne virus (arbovirus) in the Flavivirus genus of the Flaviviridae ...
... positive-sense single-stranded RNA virus of the family Flaviviridae. The hepatitis C virus particle consists of a core of ...
  • Multiple Lineages of Usutu Virus (Flaviviridae, Flavivirus) in Blackbirds (Turdus merula) and Mosquitoes (Culex pipiens, Cx. (cas.cz)
  • Zika virus (ZIKV) is a member of the Flaviviridae virus family and the Flavivirus genus. (sampspeak.in)
  • Dengue is a mosquito-borne infectious disease that affects humans and animals which is caused by the dengue virus belonging to the Flaviviridae family and genus Flavivirus. (verifiedmarketresearch.com)
  • 2019;10:753 Abstract Zika virus (ZIKV) is an arthropod-borne virus (arbovirus) in the Flavivirus genus of the Flaviviridae family. (penta-id.org)
  • Vírus Zika patrí do čeľade Flaviviridae a rodu Flavivirus (patria sem aj vírusy žltej zimnice, horúčky Dengue, Japonskej encefalitídy, Západonílskej horúčky a kliešťovej encefalitídy). (ruvzpd.sk)
  • Zika virus (ZIKV) is a member of the Flaviviridae virus own family and the Flavivirus genus, transmitted with the aid of daylight hours-lively Aedes mosquitoes, which include A. aegypti. (perfectz.net)
  • This assessment, we largely focus on zoonotic encephalitides brought on by arthropod-borne malware (arboviruses) with the families Flaviviridae (genus Flavivirus) and Togaviridae (genus Alphavirus) that are crucial in both humans and domestic family pets. (eflstudy.com)
  • Arboviruses are contained in different taxonomic families, including Flaviviridae (genus Flavivirus), Bunyaviridae (genus Nairovirus, Orthobunyavirus, Phlebovirus, and Tospovirus), Togaviridae (genus Alphavirus), Rhabdoviridae (genus Vesiculovirus), Orthomyxoviridae (genus Thogotovirus), and Reoviridae (genus Orbivirus and Coltivirus) Most of the important zoonotic arboviruses are part of the people Togaviridae and Flaviviridae [2]. (eflstudy.com)
  • Arenaviridae , Bunyaviridae , Filoviridae and Flaviviridae are all RNA viruses that can induce a coagulopathy in infected patients. (rcsi.com)
  • Guided by Monte Carlo modelling and in vitro 1-log 10 decimal-reduction value (D-value) predictions, the X-ray photon energies required for the effective inactivation of zoonotic viruses belonging to the medically important families of Flaviviridae, Nairoviridae, Phenuiviridae and Togaviridae are demonstrated. (phe.gov.uk)
  • The West Nile virus belongs to the Flaviviridae family of viruses. (ushealth.com)
  • The Hepacivirus genus comprises single-stranded positive-sense RNA viruses within the family Flaviviridae. (iec.gov.br)
  • This is because both these viruses belong to the same family called the Flaviviridae and the same mosquitoes transmit them. (top-health-today.com)
  • Dengue is a viral infection caused by four types of viruses (DENV-1, DENV-2, DENV-3, DENV-4) belonging to the Flaviviridae family. (traveldoctor.network)
  • Among the viral communities infecting Caribbean ticks, we selected four viruses belonging to the Chuviridae, Phenuiviridae and Flaviviridae families for further characterization and designing antibody screening tests. (pasteur.fr)
  • It can pass to animals and humans if they are bitten by an infected mosquito WNV is a virus of the Flaviviridae family, which includes the viruses responsible for Japanese encephalitis and dengue fever. (gdiworkathome.com)
  • Hepacivirus genus (family Flaviviridae) is an expressive example of a viral genus that grew with the advent of metagenomics. (ufrgs.br)
  • The causative factor of Zika virus disease is a virus in the family Flaviviridae spread predominantly by a mosquito in the Aedes genus. (firebaseapp.com)
  • Zika Virus infection is caused by the Zika Virus (ZIKV) belonging to the Flaviviridae family. (traveldoctor.network)
  • A digitally-colorized transmission electron micrograph of Zika virus, which is a member of the family Flaviviridae. (blogspot.com)
  • Usutu virus (USUV), a member of the mosquito-borne clade within the Flaviviridae family, was responsible for avian mortality in Austria in 2001. (elsevier.com)
  • FAO Animal hog cholera, which is caused by a Pestivirus in the Flaviviridae family. (pwnagetool-windows-live-messenger.com)
  • Hepatitis C virus is a small (55-65 nm in size), enveloped, positive-sense single-stranded RNA virus of the family Flaviviridae. (cd-diatest.com)
  • This review describes what is known about the diversity of HCV, the current classification of HCV genotypes within the family Flaviviridae and how this genetic diversity contributes to its pathogenesis. (ox.ac.uk)
  • This arbovirus belongs to Flaviviridae family and is spread to humans through the bite of an infected female Aedes aegypti mosquito and to a lesser extent from the Aedes albopictus species. (aphaih.org)
  • No Evidence of Mosquito Involvement in the Transmission of Equine Hepacivirus (Flaviviridae) in an Epidemiological Survey of Austrian Horses. (nih.gov)
  • O gênero Hepacivirus (família Flaviviridae) é um exemplo expressivo de gênero viral que cresceu rapidamente com o advento da metagenômica. (ufrgs.br)
  • Other NCPV Flaviviridae cited in literature in 2016 included NCPV 0006041v (Dengue 2 New Guinea C strain) and NCPV 0209291v (West Nile Virus NY99) that were used by scientists based in Italy, Spain and USA to validate the human enzyme DDX3 as a therapeutic target 6 . (phe-culturecollections.org.uk)

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