Electromyography
Muscle, Skeletal
Laryngeal Muscles
Facial Muscles
Biomechanical Phenomena
Neck Muscles
Fasciculation
Muscle Contraction
Neuromuscular Diseases
Neural Conduction
Shoulder
Uterine Monitoring
Computers, Analog
Voice Disorders
Temporal Muscle
Movement
Abdominal Muscles
Electrodes
Isometric Contraction
Muscle Cramp
Torque
Muscle Weakness
Musculocutaneous Nerve
Neuromuscular Agents
Muscle Fatigue
Electrodiagnosis
Chin
Man-Machine Systems
Masticatory Muscles
Deglutition
Myofunctional Therapy
Muscle Spasticity
Evoked Potentials, Motor
Laryngeal Nerves
Peripheral Nervous System Diseases
Brachial Plexus Neuropathies
Diaphragm
Biofeedback, Psychology
Reflex
Thoracic Outlet Syndrome
Lumbosacral Plexus
Reticulum
Anal Canal
Quadriceps Muscle
Neurofeedback
Cauda Equina
Back
Ulnar Nerve
Respiratory Paralysis
Pectoralis Muscles
Walking
Computer Peripherals
Botulinum Toxins, Type A
Hand
Paresis
Muscle Hypertonia
Signal Processing, Computer-Assisted
Human Engineering
Neuromuscular Nondepolarizing Agents
Accessory Nerve Injuries
Speech, Alaryngeal
Scapula
Orthotic Devices
Recruitment, Neurophysiological
Peroneal Nerve
Carpal Tunnel Syndrome
Myasthenia Gravis
Computer Terminals
Phonation
Hip
Fecal Incontinence
Range of Motion, Articular
Reflex, Stretch
Polyneuropathies
Locomotion
Pharyngeal Muscles
Reflex, Abnormal
Neuromuscular Blockade
Lifting
Monitoring, Intraoperative
Analysis of Variance
Sucking Behavior
Muscle Strength
Nerve Compression Syndromes
Paralysis
Laryngoscopy
Myography
Postural Balance
Myoclonus
Urethra
Edrophonium
Ankle Joint
Pressure
Glottis
Intercostal Nerves
Facial Nerve
Cumulative Trauma Disorders
Tendons
Muscle Rigidity
Tibial Nerve
Lower Extremity
Motor Neuron Disease
Muscle Tonus
Foot
Rectus Abdominis
Weight-Bearing
Supination
Pelvic Floor
Robotics
Median Nerve
Peripheral Nerves
Video Recording
Swimming
Voice Quality
Injections, Intramuscular
Hemiplegia
Polymyositis
Muscle Strength Dynamometer
Vocal Cords
Myofascial Pain Syndromes
Action Potentials
Vecuronium Bromide
Spasm
Androstanols
Dystonia
Spinal Cord Injuries
Electroencephalography
Myoelectric Complex, Migrating
Intercostal Muscles
Gait Disorders, Neurologic
Contracture
Electric Stimulation Therapy
Electrodes, Implanted
Spinal Nerve Roots
Task Performance and Analysis
Motor Cortex
Phrenic Nerve
Neurologic Examination
Upper Extremity
Radiculopathy
Wrist Joint
Constipation
Sural Nerve
Buttocks
Braces
Recovery of Function
Pelvis
Evoked Potentials, Somatosensory
Movement Disorders
Tremor
Evoked Potentials
Data Interpretation, Statistical
Exercise Therapy
Lumbar Vertebrae
Muscle Fibers, Skeletal
Physical Exertion
Tongue
Pain Measurement
Cerebral Palsy
Cerebellar Purkinje cell simple spike discharge encodes movement velocity in primates during visuomotor arm tracking. (1/7286)
Pathophysiological, lesion, and electrophysiological studies suggest that the cerebellar cortex is important for controlling the direction and speed of movement. The relationship of cerebellar Purkinje cell discharge to the control of arm movement parameters, however, remains unclear. The goal of this study was to examine how movement direction and speed and their interaction-velocity-modulate Purkinje cell simple spike discharge in an arm movement task in which direction and speed were independently controlled. The simple spike discharge of 154 Purkinje cells was recorded in two monkeys during the performance of two visuomotor tasks that required the animals to track targets that moved in one of eight directions and at one of four speeds. Single-parameter regression analyses revealed that a large proportion of cells had discharge modulation related to movement direction and speed. Most cells with significant directional tuning, however, were modulated at one speed, and most cells with speed-related discharge were modulated along one direction; this suggested that the patterns of simple spike discharge were not adequately described by single-parameter models. Therefore, a regression surface was fitted to the data, which showed that the discharge could be tuned to specific direction-speed combinations (preferred velocities). The overall variability in simple spike discharge was well described by the surface model, and the velocities corresponding to maximal and minimal discharge rates were distributed uniformly throughout the workspace. Simple spike discharge therefore appears to integrate information about both the direction and speed of arm movements, thereby encoding movement velocity. (+info)Spinal cord-evoked potentials and muscle responses evoked by transcranial magnetic stimulation in 10 awake human subjects. (2/7286)
Transcranial magnetic stimulation (TCMS) causes leg muscle contractions, but the neural structures in the brain that are activated by TCMS and their relationship to these leg muscle responses are not clearly understood. To elucidate this, we concomitantly recorded leg muscle responses and thoracic spinal cord-evoked potentials (SCEPs) after TCMS for the first time in 10 awake, neurologically intact human subjects. In this report we provide evidence of direct and indirect activation of corticospinal neurons after TCMS. In three subjects, SCEP threshold (T) stimulus intensities recruited both the D wave (direct activation of corticospinal neurons) and the first I wave (I1, indirect activation of corticospinal neurons). In one subject, the D, I1, and I2 waves were recruited simultaneously, and in another subject, the I1 and I2 waves were recruited simultaneously. In the remaining five subjects, only the I1 wave was recruited first. More waves were recruited as the stimulus intensity increased. The presence of D and I waves in all subjects at low stimulus intensities verified that TCMS directly and indirectly activated corticospinal neurons supplying the lower extremities. Leg muscle responses were usually contingent on the SCEP containing at least four waves (D, I1, I2, and I3). (+info)Receptor mechanisms underlying heterogenic reflexes among the triceps surae muscles of the cat. (3/7286)
The soleus (S), medial gastrocnemius (MG), and lateral gastrocnemius (LG) muscles of the cat are interlinked by rapid spinal reflex pathways. In the decerebrate state, these heterogenic reflexes are either excitatory and length dependent or inhibitory and force dependent. Mechanographic analysis was used to obtain additional evidence that the muscle spindle primary ending and the Golgi tendon organ provide the major contributions to these reflexes, respectively. The tendons of the triceps surae muscles were separated and connected to independent force transducers and servo-controlled torque motors in unanesthetized, decerebrate cats. The muscles were activated as a group using crossed-extension reflexes. Electrical stimulation of the caudal cutaneous sural nerve was used to provide a particularly strong activation of MG and decouple the forces of the triceps surae muscles. During either form of activation, the muscles were stretched either individually or in various combinations to determine the strength and characteristics of autogenic and heterogenic feedback. The corresponding force responses, including both active and passive components, were measured during the changing background tension. During activation of the entire group, the excitatory, heterogenic feedback linking the three muscles was found to be strongest onto LG and weakest onto MG, in agreement with previous results concerning the strengths of heteronymous Ia excitatory postsynaptic potentials among the triceps surae muscles. The inhibition, which is known to affect only the soleus muscle, was dependent on active contractile force and was detected essentially as rapidly as length dependent excitation. The inhibition outlasted the excitation and was blocked by intravenous strychnine. These results indicate that the excitatory and inhibitory effects are dominated by feedback from primary spindle receptors and Golgi tendon organs. The interactions between these two feedback pathways potentially can influence both the mechanical coupling between ankle and knee. (+info)Phase reversal of biomechanical functions and muscle activity in backward pedaling. (4/7286)
Computer simulations of pedaling have shown that a wide range of pedaling tasks can be performed if each limb has the capability of executing six biomechanical functions, which are arranged into three pairs of alternating antagonistic functions. An Ext/Flex pair accelerates the limb into extension or flexion, a Plant/Dorsi pair accelerates the foot into plantarflexion or dorsiflexion, and an Ant/Post pair accelerates the foot anteriorly or posteriorly relative to the pelvis. Because each biomechanical function (i.e., Ext, Flex, Plant, Dorsi, Ant, or Post) contributes to crank propulsion during a specific region in the cycle, phasing of a muscle is hypothesized to be a consequence of its ability to contribute to one or more of the biomechanical functions. Analysis of electromyogram (EMG) patterns has shown that this biomechanical framework assists in the interpretation of muscle activity in healthy and hemiparetic subjects during forward pedaling. Simulations show that backward pedaling can be produced with a phase shift of 180 degrees in the Ant/Post pair. No phase shifts in the Ext/Flex and Plant/Dorsi pairs are then necessary. To further test whether this simple yet biomechanically viable strategy may be used by the nervous system, EMGs from 7 muscles in 16 subjects were measured during backward as well as forward pedaling. As predicted, phasing in vastus medialis (VM), tibialis anterior (TA), medial gastrocnemius (MG), and soleus (SL) were unaffected by pedaling direction, with VM and SL contributing to Ext, MG to Plant, and TA to Dorsi. In contrast, phasing in biceps femoris (BF) and semimembranosus (SM) were affected by pedaling direction, as predicted, compatible with their contribution to the directionally sensitive Post function. Phasing of rectus femoris (RF) was also affected by pedaling direction; however, its ability to contribute to the directionally sensitive Ant function may only be expressed in forward pedaling. RF also contributed significantly to the directionally insensitive Ext function in both forward and backward pedaling. Other muscles also appear to have contributed to more than one function, which was especially evident in backward pedaling (i.e. , BF, SM, MG, and TA to Flex). We conclude that the phasing of only the Ant and Post biomechanical functions are directionally sensitive. Further, we suggest that task-dependent modulation of the expression of the functions in the motor output provides this biomechanics-based neural control scheme with the capability to execute a variety of lower limb tasks, including walking. (+info)Physiological properties of raphe magnus neurons during sleep and waking. (5/7286)
Neurons in the medullary raphe magnus (RM) that are important in the descending modulation of nociceptive transmission are classified by their response to noxious tail heat as ON, OFF, or NEUTRAL cells. Experiments in anesthetized animals demonstrate that RM ON cells facilitate and OFF cells inhibit nociceptive transmission. Yet little is known of the physiology of these cells in the unanesthetized animal. The first aim of the present experiments was to determine whether cells with ON- and OFF-like responses to noxious heat exist in the unanesthetized rat. Second, to determine if RM cells have state-dependent discharge, the activity of RM neurons was recorded during waking and sleeping states. Noxious heat applied during waking and slow wave sleep excited one group of cells (ON-U) in unanesthetized rats. Other cells were inhibited by noxious heat (OFF-U) applied during waking and slow wave sleep states in unanesthetized rats. NEUTRAL-U cells did not respond to noxious thermal stimulation applied during either slow wave sleep or waking. ON-U and OFF-U cells were more likely to respond to noxious heat during slow wave sleep than during waking and were least likely to respond when the animal was eating or drinking. Although RM cells rarely respond to innocuous stimulation applied during anesthesia, ON-U and OFF-U cells were excited and inhibited, respectively, by innocuous somatosensory stimulation in the unanesthetized rat. The spontaneous activity of >90% of the RM neurons recorded in the unanesthetized rat was influenced by behavioral state. OFF-U cells discharged sporadically during waking but were continuously active during slow wave sleep. By contrast, ON-U and NEUTRAL-U cells discharged in bursts during waking and either ceased to discharge entirely or discharged at a low rate during slow wave sleep. We suggest that OFF cell discharge functions to suppress pain-evoked reactions during sleep, whereas ON cell discharge facilitates pain-evoked responses during waking. (+info)Contribution of sensory feedback to the generation of extensor activity during walking in the decerebrate Cat. (6/7286)
In this investigation we have estimated the afferent contribution to the generation of activity in the knee and ankle extensor muscles during walking in decerebrate cats by loading and unloading extensor muscles, and by unilateral deafferentation of a hind leg. The total contribution of afferent feedback to extensor burst generation was estimated by allowing one hind leg to step into a hole in the treadmill belt on which the animal was walking. In the absence of ground support the level of activity in knee and ankle extensor muscles was reduced to approximately 70% of normal. Activity in the ankle extensors could be restored during the "foot-in-hole" trials by selectively resisting extension at the ankle. Thus feedback from proprioceptors in the ankle extensor muscles probably makes a large contribution to burst generation in these muscles during weight-bearing steps. Similarly, feedback from proprioceptors in knee extensor appears to contribute substantially to the activation of knee extensor muscles because unloading and loading these muscles, by lifting and dropping the hindquarters, strongly reduced and increased, respectively, the level of activity in the knee extensors. This conclusion was supported by the finding that partial deafferentation of one hind leg by transection of the L4-L6 dorsal roots reduced the level of activity in the knee extensors by approximately 50%, but did not noticeably influence the activity in ankle extensor muscles. However, extending the deafferentation to include the L7-S2 dorsal roots decreased the ankle extensor activity. We conclude that afferent feedback contributes to more than one-half of the input to knee and ankle extensor motoneurons during the stance phase of walking in decerebrate cats. The continuous contribution of afferent feedback to the generation of extensor activity could function to automatically adjust the intensity of activity to meet external demands. (+info)Visuomotor processing as reflected in the directional discharge of premotor and primary motor cortex neurons. (7/7286)
Premotor and primary motor cortical neuronal firing was studied in two monkeys during an instructed delay, pursuit tracking task. The task included a premovement "cue period," during which the target was presented at the periphery of the workspace and moved to the center of the workspace along one of eight directions at one of four constant speeds. The "track period" consisted of a visually guided, error-constrained arm movement during which the animal tracked the target as it moved from the central start box along a line to the opposite periphery of the workspace. Behaviorally, the animals tracked the required directions and speeds with highly constrained trajectories. The eye movements consisted of saccades to the target at the onset of the cue period, followed by smooth pursuit intermingled with saccades throughout the cue and track periods. Initially, an analysis of variance (ANOVA) was used to test for direction and period effects in the firing. Subsequently, a linear regression analysis was used to fit the average firing from the cue and track periods to a cosine model. Directional tuning as determined by a significant fit to the cosine model was a prominent feature of the discharge during both the cue and track periods. However, the directional tuning of the firing of a single cell was not always constant across the cue and track periods. Approximately one-half of the neurons had differences in their preferred directions (PDs) of >45 degrees between cue and track periods. The PD in the cue or track period was not dependent on the target speed. A second linear regression analysis based on calculation of the preferred direction in 20-ms bins (i.e., the PD trajectory) was used to examine on a finer time scale the temporal evolution of this change in directional tuning. The PD trajectories in the cue period were not straight but instead rotated over the workspace to align with the track period PD. Both clockwise and counterclockwise rotations occurred. The PD trajectories were relatively straight during most of the track period. The rotation and eventual convergence of the PD trajectories in the cue period to the preferred direction of the track period may reflect the transformation of visual information into motor commands. The widely dispersed PD trajectories in the cue period would allow targets to be detected over a wide spatial aperture. The convergence of the PD trajectories occurring at the cue-track transition may serve as a "Go" signal to move that was not explicitly supplied by the paradigm. Furthermore, the rotation and convergence of the PD trajectories may provide a mechanism for nonstandard mapping. Standard mapping refers to a sensorimotor transformation in which the stimulus is the object of the reach. Nonstandard mapping is the mapping of an arbitrary stimulus into an arbitrary movement. The shifts in the PD may allow relevant visual information from any direction to be transformed into an appropriate movement direction, providing a neural substrate for nonstandard stimulus-response mappings. (+info)Uncoupling of in vivo torque production from EMG in mouse muscles injured by eccentric contractions. (8/7286)
1. The main objective of this study was to determine whether eccentric contraction-induced muscle injury causes impaired plasmalemmal action potential conduction, which could explain the injury-induced excitation-contraction coupling failure. Mice were chronically implanted with stimulating electrodes on the left common peroneal nerve and with electromyographic (EMG) electrodes on the left tibialis anterior (TA) muscle. The left anterior crural muscles of anaesthetized mice were stimulated to perform 150 eccentric (ECC) (n = 12 mice) or 150 concentric (CON) (n = 11 mice) contractions. Isometric torque, EMG root mean square (RMS) and M-wave mean and median frequencies were measured before, immediately after, and at 1, 3, 5 and 14 days after the protocols. In parallel experiments, nicotinic acetylcholine receptor (AChR) concentration was measured in TA muscles to determine whether the excitation failure elicited a denervation-like response. 2. Immediately after the ECC protocol, torque was reduced by 47-89 %, while RMS was reduced by 9-21 %; the RMS decrement was not different from that observed for the CON protocol, which did not elicit large torque deficits. One day later, both ECC and CON RMS had returned to baseline values and did not change over the next 2 weeks. However, torque production by the ECC group showed a slow recovery over that time and was still depressed by 12-30 % after 2 weeks. M-wave mean and median frequencies were not affected by performance of either protocol. 3. AChR concentration was elevated by 79 and 368 % at 3 and 5 days, respectively, after the ECC protocol; AChR concentration had returned to control levels 2 weeks after the protocol. At the time of peak AChR concentration in the ECC protocol muscles (i.e. 5 days), AChR concentration in CON protocol muscles was not different from the control level. 4. In conclusion, these data demonstrate no major role for impaired plasmalemmal action potential conduction in the excitation-contraction coupling failure induced by eccentric contractions. Additionally, a muscle injured by eccentric contractions shows a response in AChR concentration similar to a transiently denervated muscle. (+info)Fasciculations can be caused by a variety of factors, including:
1. Neuronal hyperexcitability: This is the most common cause of fasciculations. It occurs when there is an imbalance in the activity of neurons in the motor unit, leading to increased excitability and muscle twitching.
2. Muscle damage: Fasciculations can occur as a result of muscle injury or strain.
3. Nutritional deficiencies: Deficiencies in vitamins such as B12 and vitamin D can cause fasciculations.
4. Medication side effects: Certain medications, such as anesthetics and anticonvulsants, can cause muscle twitching as a side effect.
5. Medical conditions: Fasciculations can be a symptom of various medical conditions, including ALS, multiple sclerosis, and peripheral neuropathy.
Fasciculations can affect any part of the body, but are most commonly seen in the eyelids, face, arms, and legs. They can be benign and temporary, or they can be a symptom of an underlying medical condition that requires treatment. If you are experiencing muscle twitching or fasciculations, it is important to speak with a healthcare professional to determine the cause and appropriate course of action.
In summary, fasciculation is a term used in neurology to describe small, localized muscle twitches that can occur in any part of the body. It can be caused by a variety of factors, including neuronal hyperexcitability, muscle damage, nutritional deficiencies, medication side effects, and medical conditions such as ALS. If you are experiencing muscle twitching or fasciculations, it is important to speak with a healthcare professional to determine the cause and appropriate course of action.
1. Muscular dystrophy: A group of genetic disorders that cause progressive muscle weakness and degeneration.
2. Amyotrophic lateral sclerosis (ALS): A progressive neurological disease that affects nerve cells in the brain and spinal cord, leading to muscle weakness, paralysis, and eventually death.
3. Spinal muscular atrophy: A genetic disorder that affects the nerve cells responsible for controlling voluntary muscle movement.
4. Peripheral neuropathy: A condition that causes damage to the peripheral nerves, leading to weakness, numbness, and pain in the hands and feet.
5. Myasthenia gravis: An autoimmune disorder that affects the nerve-muscle connection, causing muscle weakness and fatigue.
6. Neuropathy: A term used to describe damage to the nerves, which can cause a range of symptoms including numbness, tingling, and pain in the hands and feet.
7. Charcot-Marie-Tooth disease: A group of inherited disorders that affect the peripheral nerves, leading to muscle weakness and wasting.
8. Guillain-Barré syndrome: An autoimmune disorder that causes inflammation and damage to the nerves, leading to muscle weakness and paralysis.
9. Botulism: A bacterial infection that can cause muscle weakness and paralysis by blocking the release of the neurotransmitter acetylcholine.
10. Myotonia congenita: A genetic disorder that affects the nerve-muscle connection, causing muscle stiffness and rigidity.
These are just a few examples of neuromuscular diseases, and there are many more conditions that can cause muscle weakness and fatigue. It's important to see a doctor if you experience persistent or severe symptoms to receive an accurate diagnosis and appropriate treatment.
1. Muscular dystrophy: A group of genetic disorders characterized by progressive muscle weakness and degeneration.
2. Myopathy: A condition where the muscles become damaged or diseased, leading to muscle weakness and wasting.
3. Fibromyalgia: A chronic condition characterized by widespread pain, fatigue, and muscle stiffness.
4. Rhabdomyolysis: A condition where the muscle tissue is damaged, leading to the release of myoglobin into the bloodstream and potentially causing kidney damage.
5. Polymyositis/dermatomyositis: Inflammatory conditions that affect the muscles and skin.
6. Muscle strain: A common injury caused by overstretching or tearing of muscle fibers.
7. Cervical dystonia: A movement disorder characterized by involuntary contractions of the neck muscles.
8. Myasthenia gravis: An autoimmune disorder that affects the nerve-muscle connection, leading to muscle weakness and fatigue.
9. Oculopharyngeal myopathy: A condition characterized by weakness of the muscles used for swallowing and eye movements.
10. Inclusion body myositis: An inflammatory condition that affects the muscles, leading to progressive muscle weakness and wasting.
These are just a few examples of the many different types of muscular diseases that can affect individuals. Each condition has its unique set of symptoms, causes, and treatment options. It's important for individuals experiencing muscle weakness or wasting to seek medical attention to receive an accurate diagnosis and appropriate care.
Some common types of voice disorders include:
1. Dysphonia: A term used to describe difficulty speaking or producing voice sounds.
2. Aphonia: A complete loss of voice.
3. Spasmodic dysphonia: A neurological disorder characterized by involuntary movements of the vocal cords, causing a strained or breaking voice.
4. Vocal fold paralysis: A condition in which the muscles controlling the vocal cords are weakened or paralyzed, leading to a hoarse or breathy voice.
5. Vocal cord lesions: Growths, ulcers, or other injuries on the vocal cords that can affect voice quality and volume.
6. Laryngitis: Inflammation of the voice box (larynx) that can cause hoarseness and loss of voice.
7. Chronic laryngitis: A persistent form of laryngitis that can last for months or even years.
8. Acid reflux laryngitis: Gastroesophageal reflux disease (GERD) that causes stomach acid to flow up into the throat, irritating the vocal cords and causing hoarseness.
9. Vocal fold nodules: Growths on the vocal cords that can cause hoarseness and other voice changes.
10. Vocal cord polyps: Growths on the vocal cords that can cause hoarseness and other voice changes.
Voice disorders can significantly impact an individual's quality of life, as they may experience difficulty communicating effectively, loss of confidence, and emotional distress. Treatment options for voice disorders depend on the underlying cause and may include voice therapy, medications, surgery, or a combination of these approaches.
Example sentences:
1. The runner experienced a muscle cramp in her leg during the marathon, causing her to slow down and almost drop out.
2. After experiencing frequent muscle cramps, the patient was diagnosed with hypokalemia, a condition characterized by low potassium levels.
3. During pregnancy, muscle cramps are common due to changes in hormone levels and increased pressure on the musculoskeletal system.
4. The elderly man's muscle cramps were caused by a lack of physical activity and dehydration, which can be a challenge for older adults.
5. Proper stretching and warm-up exercises can help prevent muscle cramps in athletes, especially those participating in endurance sports.
There are several causes of muscle weakness, including:
1. Neuromuscular diseases: These are disorders that affect the nerves that control voluntary muscle movement, such as amyotrophic lateral sclerosis (ALS) and polio.
2. Musculoskeletal disorders: These are conditions that affect the muscles, bones, and joints, such as arthritis and fibromyalgia.
3. Metabolic disorders: These are conditions that affect the body's ability to produce energy, such as hypoglycemia and hypothyroidism.
4. Injuries: Muscle weakness can occur due to injuries such as muscle strains and tears.
5. Infections: Certain infections such as botulism and Lyme disease can cause muscle weakness.
6. Nutritional deficiencies: Deficiencies in vitamins and minerals such as vitamin D and B12 can cause muscle weakness.
7. Medications: Certain medications such as steroids and anticonvulsants can cause muscle weakness as a side effect.
The symptoms of muscle weakness can vary depending on the underlying cause, but may include:
1. Fatigue: Feeling tired or weak after performing simple tasks.
2. Lack of strength: Difficulty lifting objects or performing physical activities.
3. Muscle cramps: Spasms or twitches in the muscles.
4. Muscle wasting: Loss of muscle mass and tone.
5. Difficulty speaking or swallowing: In cases where the muscle weakness affects the face, tongue, or throat.
6. Difficulty walking or standing: In cases where the muscle weakness affects the legs or lower back.
7. Droopy facial features: In cases where the muscle weakness affects the facial muscles.
If you are experiencing muscle weakness, it is important to seek medical attention to determine the underlying cause and receive proper treatment. A healthcare professional will perform a physical examination and may order diagnostic tests such as blood tests or imaging studies to help diagnose the cause of the muscle weakness. Treatment will depend on the underlying cause, but may include medication, physical therapy, or lifestyle changes. In some cases, muscle weakness may be a sign of a serious underlying condition that requires prompt medical attention.
Dysphonia can manifest in different ways, including:
1. Hoarseness: A raspy, strained, or rough quality to the voice.
2. Breathy voice: A weak, airy, or faint voice.
3. Harsh voice: A loud, screeching, or grating voice.
4. Rough voice: A scratchy, raw, or bumpy voice.
5. Stuttering: Repetition or prolongation of sounds, syllables, or words.
6. Slurred speech: Difficulty articulating words or speaking clearly.
7. Monotone speech: Speaking in a flat, emotionless tone.
Dysphonia can be acute or chronic, and it can affect individuals of all ages and backgrounds. In some cases, dysphonia may be a symptom of an underlying medical condition, such as a viral infection, allergies, or a neurological disorder. In other cases, it may be caused by overuse or misuse of the voice, such as shouting, singing, or speaking loudly for extended periods.
Treatment options for dysphonia depend on the underlying cause and severity of the condition. Some common treatments include:
1. Voice therapy: Techniques to improve breath support, vocal technique, and speech clarity.
2. Medications: To reduce inflammation, allergies, or other underlying conditions that may be contributing to dysphonia.
3. Surgery: In some cases, surgery may be necessary to correct structural problems in the vocal cords or other areas of the voice box.
4. Laryngeal electromyography (LEMG): A test used to evaluate the function of the vocal cords and surrounding muscles.
5. Speech therapy: To improve communication skills and address any language or cognitive impairments that may be contributing to dysphonia.
6. Botulinum toxin injections (Botox): Injected into the vocal cords to reduce spasms and improve voice quality.
7. Vocal cord paralysis: In some cases, injection of a local anesthetic or botulinum toxin may be used to paralyze one or both vocal cords, allowing for rest and healing.
It's important to seek medical attention if you experience any persistent or severe changes in your voice, as early diagnosis and treatment can improve outcomes and reduce the risk of long-term vocal cord damage. A healthcare professional will be able to assess your symptoms and recommend appropriate treatment options based on the underlying cause of your dysphonia.
Muscle spasticity can cause a range of symptoms, including:
* Increased muscle tone, leading to stiffness and rigidity
* Spasms or sudden contractions of the affected muscles
* Difficulty moving the affected limbs
* Pain or discomfort in the affected area
* Abnormal postures or movements
There are several potential causes of muscle spasticity, including:
* Neurological disorders such as cerebral palsy, multiple sclerosis, and spinal cord injuries
* Stroke or other brain injuries
* Muscle damage or inflammation
* Infections such as meningitis or encephalitis
* Metabolic disorders such as hypokalemia (low potassium levels) or hyperthyroidism
Treatment options for muscle spasticity include:
* Physical therapy to improve range of motion and strength
* Medications such as baclofen, tizanidine, or dantrolene to reduce muscle spasms
* Injectable medications such as botulinum toxin or phenol to destroy excess nerve fibers
* Surgery to release or sever affected nerve fibers
* Electrical stimulation therapy to improve muscle function and reduce spasticity.
It is important to note that muscle spasticity can have a significant impact on an individual's quality of life, affecting their ability to perform daily activities, maintain independence, and engage in social and recreational activities. As such, it is important to seek medical attention if symptoms of muscle spasticity are present to determine the underlying cause and develop an appropriate treatment plan.
Peripheral Nervous System Diseases can result from a variety of causes, including:
1. Trauma or injury
2. Infections such as Lyme disease or HIV
3. Autoimmune disorders such as Guillain-Barré syndrome
4. Genetic mutations
5. Tumors or cysts
6. Toxins or poisoning
7. Vitamin deficiencies
8. Chronic diseases such as diabetes or alcoholism
Some common Peripheral Nervous System Diseases include:
1. Neuropathy - damage to the nerves that can cause pain, numbness, and weakness in the affected areas.
2. Multiple Sclerosis (MS) - an autoimmune disease that affects the CNS and PNS, causing a range of symptoms including numbness, weakness, and vision problems.
3. Peripheral Neuropathy - damage to the nerves that can cause pain, numbness, and weakness in the affected areas.
4. Guillain-Barré syndrome - an autoimmune disorder that causes muscle weakness and paralysis.
5. Charcot-Marie-Tooth disease - a group of inherited disorders that affect the nerves in the feet and legs, leading to muscle weakness and wasting.
6. Friedreich's ataxia - an inherited disorder that affects the nerves in the spine and limbs, leading to coordination problems and muscle weakness.
7. Chronic Inflammatory Demyelinating Polyneuropathy (CIDP) - an autoimmune disorder that causes inflammation of the nerves, leading to pain, numbness, and weakness in the affected areas.
8. Amyotrophic Lateral Sclerosis (ALS) - a progressive neurological disease that affects the nerve cells responsible for controlling voluntary muscle movement, leading to muscle weakness, atrophy, and paralysis.
9. Spinal Muscular Atrophy - an inherited disorder that affects the nerve cells responsible for controlling voluntary muscle movement, leading to muscle weakness and wasting.
10. Muscular Dystrophy - a group of inherited disorders that affect the nerve cells responsible for controlling voluntary muscle movement, leading to muscle weakness and wasting.
It's important to note that this is not an exhaustive list and there may be other causes of muscle weakness. If you are experiencing persistent or severe muscle weakness, it is important to see a healthcare professional for proper evaluation and diagnosis.
Brachial plexus neuropathies refer to a group of disorders that affect the brachial plexus, a network of nerves that run from the neck and shoulder down to the hand and fingers. These disorders can cause a range of symptoms including weakness, numbness, and pain in the arm and hand.
The brachial plexus is a complex network of nerves that originates in the spinal cord and branches off into several nerves that supply the shoulder, arm, and hand. Brachial plexus neuropathies can occur due to a variety of causes such as injury, trauma, tumors, cysts, infections, autoimmune disorders, and genetic mutations.
There are several types of brachial plexus neuropathies, including:
1. Erb's palsy: A condition that affects the upper roots of the brachial plexus and can cause weakness or paralysis of the arm and hand.
2. Klumpke's palsy: A condition that affects the lower roots of the brachial plexus and can cause weakness or paralysis of the hand and wrist.
3. Brachial neuritis: An inflammatory condition that causes sudden weakness and pain in the arm and hand.
4. Thoracic outlet syndrome: A condition where the nerves and blood vessels between the neck and shoulder become compressed, leading to pain and weakness in the arm and hand.
5. Neurodegenerative conditions such as amyotrophic lateral sclerosis (ALS) and peripheral neuropathy.
The symptoms of brachial plexus neuropathies can vary depending on the type and severity of the condition, but may include:
* Weakness or paralysis of the arm and hand
* Numbness or loss of sensation in the arm and hand
* Pain or aching in the arm and hand
* Muscle wasting or atrophy
* Limited range of motion in the shoulder, elbow, and wrist joints
* Decreased grip strength
* Difficulty with fine motor skills such as buttoning a shirt or tying shoelaces.
Brachial plexus neuropathies can be diagnosed through a combination of physical examination, imaging studies such as MRI or EMG, and nerve conduction studies. Treatment options vary depending on the specific condition and severity of the symptoms, but may include:
* Physical therapy to improve strength and range of motion
* Occupational therapy to improve fine motor skills and daily living activities
* Medications such as pain relievers or anti-inflammatory drugs
* Injections of corticosteroids to reduce inflammation
* Surgery to release compressed nerves or repair damaged nerve tissue.
The thoracic outlet is a narrow passageway between the scalene muscles and the first and second ribs. It contains several important structures, including the brachial plexus nerves, the subclavian artery and vein, and the phrenic nerve. When these structures are compressed or irritated, it can cause symptoms in the arm and hand.
TOS is relatively rare, but it can be caused by a variety of factors, including:
1. Congenital defects, such as a narrow thoracic outlet or abnormal development of the rib cage.
2. Trauma, such as a fall onto the shoulder or a direct blow to the chest.
3. Repetitive movements, such as typing or using a computer mouse.
4. Poor posture or body mechanics.
5. Muscle imbalances or weakness in the neck and shoulder muscles.
6. Ganglion cysts or other soft tissue masses that compress the nerves or blood vessels.
7. Fractures or dislocations of the clavicle or shoulder blade.
8. Tumors or other abnormal growths in the chest or neck.
9. Inflammatory conditions, such as rheumatoid arthritis or thyroiditis.
Symptoms of TOS can vary depending on the location and severity of the compression. They may include:
1. Pain in the shoulder or arm, which can be exacerbated by movement or activity.
2. Numbness, tingling, or weakness in the hand or fingers.
3. Difficulty coordinating movements or performing fine motor tasks.
4. Weakness or fatigue in the muscles of the shoulder and arm.
5. Decreased grip strength or dexterity.
6. Pain or tingling that radiates down the arm or into the hand.
7. Swelling or redness in the neck or shoulder.
8. Difficulty swallowing or breathing, in severe cases.
TOS can be difficult to diagnose, as the symptoms can be similar to those of other conditions such as carpal tunnel syndrome or a heart attack. A thorough physical examination and medical history are important for making an accurate diagnosis. Imaging studies such as X-rays, CT scans, or MRI may also be used to help identify any underlying structural abnormalities or nerve compression. Electromyography (EMG) and nerve conduction studies (NCS) may also be performed to assess nerve function and determine the extent of nerve damage.
Treatment for TOS depends on the underlying cause and severity of the condition. Conservative treatments may include:
1. Rest and avoidance of activities that exacerbate the symptoms.
2. Physical therapy to improve posture, strength, and range of motion.
3. Anti-inflammatory medications or pain relievers to reduce swelling and relieve pain.
4. Muscle relaxants to reduce muscle spasm and tension.
5. Injections of steroids or local anesthetics to reduce inflammation and relieve pain.
6. Surgery may be necessary in severe cases, such as when there is significant nerve compression or instability of the shoulder joint.
It's important to seek medical attention if you experience any symptoms of TOS, as early diagnosis and treatment can help prevent long-term complications and improve outcomes.
Respiratory paralysis can manifest in different ways depending on the underlying cause and severity of the condition. Some common symptoms include:
1. Difficulty breathing: Patients may experience shortness of breath, wheezing, or a feeling of suffocation.
2. Weakened cough reflex: The muscles used for coughing may be weakened or paralyzed, making it difficult to clear secretions from the lungs.
3. Fatigue: Breathing can be tiring and may leave the patient feeling exhausted.
4. Sleep disturbances: Respiratory paralysis can disrupt sleep patterns and cause insomnia or other sleep disorders.
5. Chest pain: Pain in the chest or ribcage can be a symptom of respiratory paralysis, particularly if it is caused by muscle weakness or atrophy.
Diagnosis of respiratory paralysis typically involves a physical examination, medical history, and diagnostic tests such as electroencephalogram (EEG), electromyography (EMG), or nerve conduction studies (NCS). Treatment options vary depending on the underlying cause but may include:
1. Medications: Drugs such as bronchodilators, corticosteroids, and anticholinergics can be used to manage symptoms and improve lung function.
2. Respiratory therapy: Techniques such as chest physical therapy, respiratory exercises, and non-invasive ventilation can help improve lung function and reduce fatigue.
3. Surgery: In some cases, surgery may be necessary to correct anatomical abnormalities or repair damaged nerves.
4. Assistive devices: Patients with severe respiratory paralysis may require the use of assistive devices such as oxygen therapy, ventilators, or wheelchairs to help improve their quality of life.
5. Rehabilitation: Physical therapy, occupational therapy, and speech therapy can all be helpful in improving function and reducing disability.
6. Lifestyle modifications: Patients with respiratory paralysis may need to make lifestyle changes such as avoiding smoke, dust, and other irritants, getting regular exercise, and managing stress to help improve their condition.
The symptoms of paresis may include weakness or paralysis of specific muscle groups, loss of sensation, tremors, and difficulty with coordination and balance. The severity of the paresis can vary depending on the underlying cause and the extent of the damage to the nervous system. Treatment options for paresis depend on the underlying cause and may include physical therapy, medications, surgery, or other interventions aimed at improving motor function and preventing complications.
In summary, paresis is a loss or impairment of motor function resulting from damage to the nervous system, and can be caused by various conditions such as stroke, traumatic brain injury, and neurological disorders. Treatment options depend on the underlying cause and may include physical therapy, medications, surgery, or other interventions aimed at improving motor function and preventing complications.
There are several potential causes of muscle hypertonia, including:
1. Neurological disorders such as cerebral palsy, Parkinson's disease, or multiple sclerosis
2. Musculoskeletal injuries or inflammation
3. Infections such as Lyme disease or viral infections
4. Metabolic disorders such as hypokalemia (low potassium levels) or hyperthyroidism
5. Adverse reactions to certain medications
6. Emotional stress or anxiety
Symptoms of muscle hypertonia can vary depending on the severity and location of the condition, but may include:
1. Stiffness and rigidity of the affected muscles
2. Pain or tenderness in the muscles
3. Limited range of motion in the affected joints
4. Fatigue or weakness in the affected limbs
5. Difficulty with movement and balance
6. Muscle spasms or cramping
Treatment for muscle hypertonia typically involves a combination of physical therapy, medication, and lifestyle modifications. Physical therapy may include stretching and strengthening exercises to improve range of motion and reduce stiffness, as well as techniques such as heat or cold therapy to relax the muscles. Medications such as muscle relaxants or anti-inflammatory drugs may be prescribed to reduce muscle spasms and inflammation. Lifestyle modifications such as regular exercise, proper nutrition, and stress management techniques can also help to reduce symptoms of muscle hypertonia. In severe cases, surgery may be necessary to release or lengthen the affected muscles.
Injuries to the accessory nerve can be caused by a variety of factors, including:
Trauma: Trauma to the neck or shoulder region can cause injury to the accessory nerve, leading to weakness or paralysis of the sternocleidomastoid muscle and other muscles of the neck and shoulder girdle.
Surgery or other medical procedures: Accessory nerve injuries can occur as a complication of surgery or other medical procedures in the neck or shoulder region, such as thyroid or parathyroid surgery, laryngectomy, or cervical spine surgery.
Infections: Infections such as meningitis or abscesses can cause inflammation and damage to the accessory nerve, leading to injury.
Tumors: Tumors in the neck or shoulder region can compress or damage the accessory nerve, leading to weakness or paralysis of the sternocleidomastoid muscle and other muscles of the neck and shoulder girdle.
Congenital conditions: Some congenital conditions, such as Turner syndrome or Down syndrome, can affect the development of the accessory nerve and cause weakness or paralysis of the sternocleidomastoid muscle and other muscles of the neck and shoulder girdle.
Symptoms of accessory nerve injuries may include:
Weakness or paralysis of the sternocleidomastoid muscle and other muscles of the neck and shoulder girdle, which can cause difficulty swallowing, breathing, or moving the head and neck.
Pain in the neck or shoulder region, which may be severe and persistent.
Numbness or tingling sensations in the neck, shoulder, or face.
Difficulty speaking or swallowing, which can affect communication and eating.
Weakness or paralysis of other muscles that are innervated by the accessory nerve, such as the trapezius or deltoid muscles.
Trouble moving the arm or hand, which can affect daily activities and functionality.
If you suspect that you have an accessory nerve injury, it is important to seek medical attention as soon as possible. Your healthcare provider will perform a physical examination and may order imaging studies, such as X-rays, CT scans, or MRI scans, to determine the extent of the injury and identify any underlying causes. Treatment for accessory nerve injuries may include:
Physical therapy to improve strength and range of motion in the affected muscles.
Medications, such as pain relievers, anti-inflammatory drugs, or muscle relaxants, to manage symptoms.
Surgery to repair or release compressed or damaged nerve tissue.
Injections of steroids or other medications to reduce inflammation and promote healing.
It is important to seek medical attention if you experience any signs or symptoms of an accessory nerve injury, as prompt treatment can help to prevent long-term complications and improve outcomes.
* Numbness or tingling in the fingers and thumb, especially the index and middle fingers
* Pain in the wrist, hand, or fingers
* Weakness in the hand, making it difficult to grip or hold objects
* Tingling or burning sensations in the fingers and thumb
* Loss of dexterity and coordination in the hand
CTS can be caused by a variety of factors, including:
* Repetitive motion, such as typing or using a computer mouse for long periods of time
* Injury to the wrist or hand
* Fluid retention during pregnancy or menopause
* Anatomical variations, such as a narrower carpal tunnel or a thicker median nerve
* Other medical conditions, such as diabetes, thyroid disorders, or rheumatoid arthritis
Treatment for CTS can range from conservative methods, such as physical therapy, splints, and medication, to surgical intervention. It is important to seek medical attention if symptoms persist or worsen over time, as untreated CTS can lead to permanent nerve damage and disability.
The symptoms of myasthenia gravis can vary in severity and may include:
* Weakness in the arms and legs
* Fatigue and muscle tiredness
* Difficulty swallowing (dysphagia)
* Difficulty speaking or slurred speech (dysarthria)
* Drooping eyelids (ptosis)
* Double vision (diplopia)
* Weakness in the muscles of the face, arms, and legs
The exact cause of myasthenia gravis is not known, but it is believed to be an autoimmune disorder, meaning that the body's immune system mistakenly attacks healthy tissues. It can also be caused by other medical conditions such as thyroid disease, vitamin deficiencies, or infections.
There is no cure for myasthenia gravis, but there are various treatments available to manage the symptoms and improve quality of life. These include:
* Medications such as corticosteroids, immunosuppressants, and cholinesterase inhibitors
* Plasmapheresis, a procedure that removes harmful antibodies from the blood
* Intravenous immunoglobulin (IVIG), which contains antibodies that can help block the immune system's attack on the nerve-muscle junction
* Surgery to remove the thymus gland, which is believed to play a role in the development of myasthenia gravis
It is important for individuals with myasthenia gravis to work closely with their healthcare provider to manage their symptoms and prevent complications. With proper treatment and self-care, many people with myasthenia gravis are able to lead active and fulfilling lives.
The causes of FI can be classified into two main categories: anorectal mechanical disorders and neurological disorders. Anorectal mechanical disorders include conditions such as rectocele, rectal prolapse, and anal sphincter dysfunction. Neurological disorders include conditions such as spinal cord injuries, multiple sclerosis, and Parkinson's disease.
Symptoms of FI may include:
* Involuntary passage of stool
* Straining during defecation
* Lack of sensation during defecation
* Incomplete evacuation of stool
* Anal itching or irritation
The diagnosis of FI typically involves a comprehensive medical history, physical examination, and various tests such as anorectal manometry, endoanal ultrasonography, and balloon expulsion tests. Treatment options for FI depend on the underlying cause and severity of symptoms, but may include:
* Dietary modifications
* Biofeedback therapy
* Pelvic floor exercises (Kegel exercises)
* Anorectal surgery
* Stool softeners or laxatives
* Anal plugs or suppositories
It is important to note that FI can have a significant impact on an individual's quality of life, and it is essential to seek medical attention if symptoms persist or worsen over time. With proper diagnosis and treatment, many individuals with FI are able to experience improved symptoms and a better quality of life.
There are many different causes of polyneuropathy, including:
1. Diabetes: High blood sugar levels over time can damage nerves, leading to numbness, tingling, and pain in the hands and feet.
2. Vitamin deficiencies: Deficiencies in vitamins such as B12 and B6 can cause nerve damage and polyneuropathy.
3. Toxins: Exposure to certain toxins, such as heavy metals or pesticides, can damage nerves and cause polyneuropathy.
4. Infections: Certain infections, such as Lyme disease and HIV, can cause polyneuropathy.
5. Autoimmune disorders: Conditions such as Guillain-Barré syndrome and chronic inflammatory demyelinating polyneuropathy (CIDP) are autoimmune disorders that can cause polyneuropathy.
6. Trauma: Physical trauma, such as a severe injury or crush injury, can cause polyneuropathy.
7. Cancer: Certain types of cancer, such as lymphoma and leukemia, can cause polyneuropathy.
8. Genetic disorders: Some inherited conditions, such as Charcot-Marie-Tooth disease, can cause polyneuropathy.
The symptoms of polyneuropathy depend on the specific nerves affected and can include:
1. Numbness or tingling in the hands and feet
2. Pain in the hands and feet
3. Weakness in the muscles of the hands and feet
4. Difficulty walking or maintaining balance
5. Loss of reflexes
6. Sensitivity to touch or temperature changes
7. Muscle wasting
8. Decreased dexterity
9. Tremors
10. Autonomic dysfunction (e.g., bowel or bladder problems)
The diagnosis of polyneuropathy is based on a combination of clinical findings, nerve conduction studies, and laboratory tests. Treatment depends on the underlying cause of the condition and may include:
1. Pain management with medications such as pain relievers or anti-seizure drugs
2. Physical therapy to maintain muscle strength and mobility
3. Occupational therapy to improve daily functioning
4. Assistive devices, such as canes or walkers, to aid with mobility
5. Autonomic dysfunction management with medications such as beta blockers or fludrocortisone
6. Plasmapheresis, a procedure that removes harmful antibodies from the blood
7. Immunoglobulin therapy, which can help to reduce inflammation
8. Intravenous immunoglobulin (IVIG) therapy, which can help to reduce inflammation and repair nerve damage
9. Dietary changes, such as increasing protein intake, to support nerve health
10. Avoiding harmful substances, such as alcohol or tobacco, which can worsen the condition.
Examples of abnormal reflexes include:
1. Overactive reflexes: Reflexes that are too strong or exaggerated, such as an oversensitive knee jerk reflex.
2. Underactive reflexes: Reflexes that are too weak or diminished, such as a decreased tendon reflex in the arm.
3. Delayed reflexes: Reflexes that take longer than expected to occur, such as a delayed deep tendon reflex.
4. Abnormal reflex arc: A reflex arc that is not normal or expected for the situation, such as a spastic reflex arc.
5. Reflexes that are out of proportion to the stimulus: Such as an excessive or exaggerated reflex response to a mild stimulus.
6. Reflexes that occur in the absence of a stimulus: Such as a spontaneous reflex.
7. Reflexes that do not resolve: Such as a persistent reflex.
8. Reflexes that are painful or uncomfortable: Such as an abnormal rectal reflex.
It's important to note that not all abnormal reflexes are necessarily indicative of a serious medical condition, but they should be evaluated by a healthcare professional to determine the underlying cause and appropriate treatment.
There are several types of nerve compression syndromes, including:
1. Carpal tunnel syndrome: Compression of the median nerve in the wrist, commonly caused by repetitive motion or injury.
2. Tarsal tunnel syndrome: Compression of the posterior tibial nerve in the ankle, similar to carpal tunnel syndrome but affecting the lower leg.
3. Cubital tunnel syndrome: Compression of the ulnar nerve at the elbow, often caused by repetitive leaning or bending.
4. Thoracic outlet syndrome: Compression of the nerves and blood vessels that pass through the thoracic outlet (the space between the neck and shoulder), often caused by poor posture or injury.
5. Peripheral neuropathy: A broader term for damage to the peripheral nerves, often caused by diabetes, vitamin deficiencies, or other systemic conditions.
6. Meralgia paresthetica: Compression of the lateral femoral cutaneous nerve in the thigh, commonly caused by direct trauma or compression from a tight waistband or clothing.
7. Morton's neuroma: Compression of the plantar digital nerves between the toes, often caused by poorly fitting shoes or repetitive stress on the feet.
8. Neuralgia: A general term for pain or numbness caused by damage or irritation to a nerve, often associated with chronic conditions such as shingles or postherpetic neuralgia.
9. Trigeminal neuralgia: A condition characterized by recurring episodes of sudden, extreme pain in the face, often caused by compression or irritation of the trigeminal nerve.
10. Neuropathic pain: Pain that occurs as a result of damage or dysfunction of the nervous system, often accompanied by other symptoms such as numbness, tingling, or weakness.
1. Complete paralysis: When there is no movement or sensation in a particular area of the body.
2. Incomplete paralysis: When there is some movement or sensation in a particular area of the body.
3. Localized paralysis: When paralysis affects only a specific part of the body, such as a limb or a facial muscle.
4. Generalized paralysis: When paralysis affects multiple parts of the body.
5. Flaccid paralysis: When there is a loss of muscle tone and the affected limbs feel floppy.
6. Spastic paralysis: When there is an increase in muscle tone and the affected limbs feel stiff and rigid.
7. Paralysis due to nerve damage: This can be caused by injuries, diseases such as multiple sclerosis, or birth defects such as spina bifida.
8. Paralysis due to muscle damage: This can be caused by injuries, such as muscular dystrophy, or diseases such as muscular sarcopenia.
9. Paralysis due to brain damage: This can be caused by head injuries, stroke, or other conditions that affect the brain such as cerebral palsy.
10. Paralysis due to spinal cord injury: This can be caused by trauma, such as a car accident, or diseases such as polio.
Paralysis can have a significant impact on an individual's quality of life, affecting their ability to perform daily activities, work, and participate in social and recreational activities. Treatment options for paralysis depend on the underlying cause and may include physical therapy, medications, surgery, or assistive technologies such as wheelchairs or prosthetic devices.
Myoclonus can be classified into several types based on its duration, frequency, and distribution. Some common types of myoclonus include:
1. Generalized myoclonus: This type affects the entire body and is often seen in conditions such as epilepsy, encephalitis, and multiple sclerosis.
2. Localized myoclonus: This type affects a specific area of the body, such as the arm or leg.
3. Progressive myoclonus: This type worsens over time and is often seen in conditions such as Parkinson's disease and Huntington's disease.
4. Periodic myoclonus: This type is characterized by recurring episodes of muscle contractions and releases, often triggered by specific stimuli such as noise or stress.
5. Task-specific myoclonus: This type is seen in individuals who perform repetitive tasks, such as typing or using a computer mouse.
Myoclonus can cause a range of symptoms, including muscle weakness, fatigue, and difficulty with coordination and balance. In some cases, myoclonus can also lead to falls or injuries. Treatment for myoclonus depends on the underlying cause and may include medications such as anticonvulsants, physical therapy, and lifestyle modifications.
Myoclonus is a relatively rare condition, but it can have a significant impact on an individual's quality of life. It can affect their ability to perform daily activities, participate in social events, and maintain their independence. If you or someone you know has been diagnosed with myoclonus, it is important to work closely with a healthcare provider to develop a personalized treatment plan and manage the condition effectively.
Types of Peripheral Nerve Injuries:
1. Traumatic Nerve Injury: This type of injury occurs due to direct trauma to the nerve, such as a blow or a crush injury.
2. Compression Neuropathy: This type of injury occurs when a nerve is compressed or pinched, leading to damage or disruption of the nerve signal.
3. Stretch Injury: This type of injury occurs when a nerve is stretched or overstretched, leading to damage or disruption of the nerve signal.
4. Entrapment Neuropathy: This type of injury occurs when a nerve is compressed or trapped between two structures, leading to damage or disruption of the nerve signal.
Symptoms of Peripheral Nerve Injuries:
1. Weakness or paralysis of specific muscle groups
2. Numbness or tingling in the affected area
3. Pain or burning sensation in the affected area
4. Difficulty with balance and coordination
5. Abnormal reflexes
6. Incontinence or other bladder or bowel problems
Causes of Peripheral Nerve Injuries:
1. Trauma, such as a car accident or fall
2. Sports injuries
3. Repetitive strain injuries, such as those caused by repetitive motions in the workplace or during sports activities
4. Compression or entrapment of nerves, such as carpal tunnel syndrome or tarsal tunnel syndrome
5. Infections, such as Lyme disease or diphtheria
6. Tumors or cysts that compress or damage nerves
7. Vitamin deficiencies, such as vitamin B12 deficiency
8. Autoimmune disorders, such as rheumatoid arthritis or lupus
9. Toxins, such as heavy metals or certain chemicals
Treatment of Peripheral Nerve Injuries:
1. Physical therapy to improve strength and range of motion
2. Medications to manage pain and inflammation
3. Surgery to release compressed nerves or repair damaged nerves
4. Electrical stimulation therapy to promote nerve regeneration
5. Platelet-rich plasma (PRP) therapy to stimulate healing
6. Stem cell therapy to promote nerve regeneration
7. Injection of botulinum toxin to relieve pain and reduce muscle spasticity
8. Orthotics or assistive devices to improve mobility and function
It is important to seek medical attention if you experience any symptoms of a peripheral nerve injury, as early diagnosis and treatment can help prevent long-term damage and improve outcomes.
The term "cumulative" refers to the gradual buildup of damage over time, as opposed to a single traumatic event that causes immediate harm. The damage can result from repetitive motions, vibrations, compressive forces, or other forms of stress that accumulate and lead to tissue injury and inflammation.
Some common examples of CTDs include:
1. Carpal tunnel syndrome: A condition that affects the wrist and hand, caused by repetitive motion and compression of the median nerve.
2. Tendinitis: Inflammation of a tendon, often caused by repetitive motion or overuse.
3. Bursitis: Inflammation of a bursa, a fluid-filled sac that cushions joints and reduces friction between tissues.
4. Tennis elbow: A condition characterized by inflammation of the tendons on the outside of the elbow, caused by repetitive gripping or twisting motions.
5. Plantar fasciitis: Inflammation of the plantar fascia, a band of tissue that runs along the bottom of the foot, caused by repetitive strain and overuse.
6. Repetitive stress injuries: A broad category of injuries caused by repetitive motion, such as typing or using a computer mouse.
7. Occupational asthma: A condition caused by inhaling allergens or irritants in the workplace, leading to inflammation and narrowing of the airways.
8. Hearing loss: Damage to the inner ear or auditory nerve caused by exposure to loud noises over time.
9. Vibration white finger: A condition that affects the hands, causing whiteness or loss of blood flow in the fingers due to exposure to vibrating tools.
10. Carpal tunnel syndrome: Compression of the median nerve in the wrist, leading to numbness, tingling, and weakness in the hand and arm.
It's important to note that these conditions can have a significant impact on an individual's quality of life, ability to work, and overall well-being. If you are experiencing any of these conditions, it is important to seek medical attention to receive proper diagnosis and treatment.
There are several possible causes of muscle rigidity, including:
1. Injury: Muscle rigidity can be a result of direct trauma to the muscle, such as a strain or sprain.
2. Infection: Certain infections, such as Lyme disease or endocarditis, can cause muscle rigidity as a symptom.
3. Neurological disorders: Conditions such as multiple sclerosis, Parkinson's disease, and stroke can all cause muscle rigidity due to damage to the nervous system.
4. Medication side effects: Certain medications, such as steroids and certain antidepressants, can cause muscle rigidity as a side effect.
5. Metabolic disorders: Conditions such as hypocalcemia (low calcium levels) and hyperthyroidism can cause muscle rigidity.
6. Autoimmune disorders: Conditions such as polymyositis and dermatomyositis can cause muscle rigidity due to inflammation of the muscles.
Symptoms of muscle rigidity may include:
* Stiffness or inflexibility in the affected muscles
* Pain or tenderness in the affected area
* Limited range of motion in the affected joints
* Muscle spasms or cramps
* Fatigue or weakness
Treatment for muscle rigidity will depend on the underlying cause. In some cases, medication may be prescribed to relax the muscles and improve mobility. Physical therapy and exercise may also be helpful in improving range of motion and strength. In other cases, treatment may involve addressing the underlying condition or disorder that is causing the muscle rigidity.
MND is often fatal, usually within 2-5 years of diagnosis. There is currently no cure for MND, although various treatments and therapies can help manage the symptoms and slow its progression.
The most common types of MND are amyotrophic lateral sclerosis (ALS) and primary lateral sclerosis (PLS). ALS is characterized by rapid degeneration of motor neurons in the brain and spinal cord, leading to muscle weakness and paralysis. PLS is a slower-progressing form of MND that affects only the lower motor neurons.
MND can be caused by a variety of factors, including genetics, age, and exposure to toxins. It is often diagnosed through a combination of medical history, physical examination, and diagnostic tests such as electromyography (EMG) and magnetic resonance imaging (MRI).
There is ongoing research into the causes and potential treatments for MND, including stem cell therapy, gene therapy, and drugs that target specific molecules involved in the disease process.
Blepharospasm is a type of movement disorder that affects the eyelids, causing them to twitch or spasm involuntarily. The condition can be caused by a variety of factors, including:
1. Stress and fatigue: High levels of stress and fatigue can lead to muscle tension in the eyelids, resulting in blepharospasm.
2. Caffeine withdrawal: Suddenly stopping or reducing caffeine intake can cause withdrawal symptoms, including blepharospasm.
3. Medications: Certain medications, such as antidepressants and antipsychotics, can cause blepharospasm as a side effect.
4. Neurological disorders: In some cases, blepharospasm may be a symptom of an underlying neurological disorder, such as dystonia or Parkinson's disease.
5. Other causes: Blepharospasm can also be caused by other factors, such as dry eyes, allergies, or exposure to bright lights.
Treatment options for blepharospasm include:
1. Relaxation techniques: Techniques such as deep breathing, progressive muscle relaxation, and visualization can help reduce stress and muscle tension in the eyelids.
2. Botulinum toxin injections: Injecting botulinum toxin into the eyelid muscles can weaken the muscles and reduce the frequency and severity of blepharospasm.
3. Surgery: In severe cases of blepharospasm, surgery may be necessary to remove part of the affected muscle or to alter the position of the eyelid.
4. Medications: Various medications, such as anticholinergic drugs and benzodiazepines, can help reduce the symptoms of blepharospasm.
5. Glasses or contact lenses: In some cases, wearing glasses or contact lenses may help reduce the symptoms of blepharospasm by reducing glare and improving vision.
It is important to note that the best course of treatment will depend on the underlying cause of the blepharospasm, and a healthcare professional should be consulted to determine the appropriate treatment plan.
Hemiplegia can cause a range of symptoms including weakness, paralysis, loss of sensation, and difficulty with movement and coordination on one side of the body. The affected side may also experience muscle spasticity or rigidity, causing stiffness and limited mobility.
Depending on the severity and location of the damage, hemiplegia can be classified into different types:
1. Left hemiplegia: This type affects the left side of the body and is caused by damage to the left hemisphere of the brain.
2. Right hemiplegia: This type affects the right side of the body and is caused by damage to the right hemisphere of the brain.
3. Mixed hemiplegia: This type affects both sides of the body and is caused by damage to both hemispheres of the brain or other areas of the brainstem.
4. Progressive hemiplegia: This type progressively worsens over time and is often associated with neurodegenerative disorders such as Parkinson's disease or multiple sclerosis.
Treatment for hemiplegia typically focuses on physical therapy, occupational therapy, and rehabilitation to improve mobility, strength, and function. Medications such as anticonvulsants, muscle relaxants, and pain relievers may also be prescribed to manage symptoms. In severe cases, surgery may be necessary to relieve pressure on the brain or spinal cord.
In summary, hemiplegia is a condition characterized by paralysis or weakness on one side of the body, often caused by damage to the brain or spinal cord. Treatment options vary depending on the severity and underlying cause of the condition.
Polymyositis can affect people of all ages, but it most commonly occurs in adults between the ages of 30 and 60. It is more common in women than men, and the symptoms can vary in severity. The disease may be acute or chronic, and it can affect one or more muscle groups.
The symptoms of polymyositis include:
* Muscle weakness and fatigue
* Pain in the affected muscles
* Wasting of the affected muscles
* Difficulty swallowing (in severe cases)
* Shortness of breath (in severe cases)
The diagnosis of polymyositis is based on a combination of clinical findings, laboratory tests, and imaging studies. Laboratory tests may include blood tests to check for muscle enzymes and inflammatory markers, such as erythrocyte sedimentation rate (ESR) and C-reactive protein (CRP). Imaging studies, such as magnetic resonance imaging (MRI), can help to confirm the diagnosis and assess the extent of the disease.
There is no cure for polymyositis, but treatment can help to manage the symptoms and slow the progression of the disease. Treatment options may include:
* Corticosteroids to reduce inflammation
* Immunosuppressive drugs to suppress the immune system
* Physical therapy to maintain muscle strength and function
* Pain management with analgesics and other medications
* Plasmapheresis to remove antibodies from the blood
The prognosis for polymyositis varies, depending on the severity of the disease and the response to treatment. In general, the prognosis is better for patients who have a mild form of the disease and who respond well to treatment. However, in severe cases, the disease can be life-threatening, and mortality rates are estimated to be as high as 20% to 30%.
Symptoms of myofascial pain syndrome include:
* Pain in specific areas of the body, such as the neck, back, or limbs
* Pain that is worse with movement or activity
* Muscle stiffness and limited range of motion
* Trigger points, which are areas of hypersensitivity within the muscle that can cause pain when stimulated
* Poor posture or gait
* Fatigue
* Decreased strength and endurance
Treatment for myofascial pain syndrome typically involves a combination of physical therapy, pain management strategies, and self-care techniques. Physical therapy may include stretching exercises, myofascial release techniques, and other modalities to help relieve pain and improve range of motion. Pain management strategies may include medication, injections, or alternative therapies such as acupuncture or massage. Self-care techniques can also be helpful, such as heat or cold applications, relaxation techniques, and good posture.
The prognosis for myofascial pain syndrome varies depending on the severity of the condition and the effectiveness of treatment. In general, with appropriate treatment and self-care, many people are able to manage their symptoms and improve their quality of life. However, in some cases, the condition can be challenging to treat and may require ongoing management.
Overall, myofascial pain syndrome is a common and often misunderstood condition that can cause significant pain and disability. With proper diagnosis and treatment, however, many people are able to find relief and improve their quality of life.
Example sentences:
1. The patient experienced a spasm in their leg while running, causing them to stumble and fall.
2. The doctor diagnosed the patient with muscle spasms caused by dehydration and recommended increased fluids and stretching exercises.
3. The athlete suffered from frequent leg spasms during their training, which affected their performance and required regular massage therapy to relieve the discomfort.
Some common forms of dystonia include:
1. Generalized dystonia: This is the most common form of dystonia, affecting the entire body.
2. Focal dystonia: This type affects only one part of the body, such as the hand or foot.
3. Task-specific dystonia: This type is caused by a specific activity or task, such as writing or playing a musical instrument.
4. Torticollis: This is a type of dystonia that affects the neck and causes it to twist or tilts to one side.
5. Blepharospasm: This is a type of dystonia that affects the eyelids, causing them to spasm or twitch.
6. Oromandibular dystonia: This type affects the muscles of the face and jaw, causing unusual movements of the mouth and tongue.
7. Meige syndrome: This is a rare form of dystonia that affects both the eyes and the eyelids, causing them to twitch or spasm.
The symptoms of dystonia can vary depending on the type and severity of the disorder. They may include:
* Involuntary muscle contractions or spasms
* Twisting or repetitive movements of the affected body part
* Pain or discomfort in the affected area
* Difficulty with movement or coordination
* Fatigue or weakness
* Cramps or spasms
Dystonia can be caused by a variety of factors, including:
* Genetic mutations: Many forms of dystonia are inherited, and they can be caused by mutations in specific genes.
* Brain injury: Dystonia can be caused by a head injury or other trauma to the brain.
* Infections: Certain infections, such as encephalitis or meningitis, can cause dystonia.
* Stroke or other vascular conditions: A stroke or other conditions that affect blood flow to the brain can cause dystonia.
* Neurodegenerative diseases: Dystonia can be a symptom of neurodegenerative diseases such as Parkinson's disease, Huntington's disease, or progressive supranuclear palsy.
There is no cure for dystonia, but there are several treatment options available to help manage the symptoms. These may include:
* Medications: Injectable drugs such as botulinum toxin (Botox) or oral medications such as anticholinergic agents can help relax the muscles and reduce spasms.
* Physical therapy: Physical therapy exercises can help improve movement and coordination, and reduce muscle stiffness.
* Speech therapy: For people with dystonia affecting the face or tongue, speech therapy may be helpful in improving communication and addressing swallowing difficulties.
* Surgery: In some cases, surgery may be necessary to relieve symptoms. This can involve cutting or destroying certain muscles or nerves that are causing the dystonia.
* Deep brain stimulation: A procedure in which an electrode is implanted in the brain to deliver electrical impulses to specific areas of the brain, this can help reduce symptoms in some people with dystonia.
It's important to note that each person with dystonia is unique and may respond differently to different treatments. A healthcare professional will work with the individual to develop a personalized treatment plan that takes into account their specific needs and symptoms.
There are several different types of spinal cord injuries that can occur, depending on the location and severity of the damage. These include:
1. Complete spinal cord injuries: In these cases, the spinal cord is completely severed, resulting in a loss of all sensation and function below the level of the injury.
2. Incomplete spinal cord injuries: In these cases, the spinal cord is only partially damaged, resulting in some remaining sensation and function below the level of the injury.
3. Brown-Sequard syndrome: This is a specific type of incomplete spinal cord injury that affects one side of the spinal cord, resulting in weakness or paralysis on one side of the body.
4. Conus medullaris syndrome: This is a type of incomplete spinal cord injury that affects the lower part of the spinal cord, resulting in weakness or paralysis in the legs and bladder dysfunction.
The symptoms of spinal cord injuries can vary depending on the location and severity of the injury. They may include:
* Loss of sensation in the arms, legs, or other parts of the body
* Weakness or paralysis in the arms, legs, or other parts of the body
* Difficulty walking or standing
* Difficulty with bowel and bladder function
* Numbness or tingling sensations
* Pain or pressure in the neck or back
Treatment for spinal cord injuries typically involves a combination of medical and rehabilitative therapies. Medical treatments may include:
* Immobilization of the spine to prevent further injury
* Medications to manage pain and inflammation
* Surgery to relieve compression or stabilize the spine
Rehabilitative therapies may include:
* Physical therapy to improve strength and mobility
* Occupational therapy to learn new ways of performing daily activities
* Speech therapy to improve communication skills
* Psychological counseling to cope with the emotional effects of the injury.
Overall, the prognosis for spinal cord injuries depends on the severity and location of the injury, as well as the age and overall health of the individual. While some individuals may experience significant recovery, others may experience long-term or permanent impairment. It is important to seek medical attention immediately if symptoms of a spinal cord injury are present.
The symptoms of gait disorders, neurologic can vary depending on the underlying cause, but may include:
* Difficulty walking or standing
* Ataxia (loss of coordination)
* Spasticity (stiffness) or rigidity (inflexibility)
* Bradykinesia (slowness of movement)
* Scanning (looking for support while walking)
* Pauses or freezing during gait
* Loss of balance or poor equilibrium
* Increased risk of falling
Gait disorders, neurologic can have a significant impact on an individual's quality of life, as they may limit their ability to perform daily activities and increase their risk of falling. Treatment for these disorders typically involves a combination of physical therapy, occupational therapy, and medications to manage symptoms such as spasticity and bradykinesia. In some cases, surgery or other interventions may be necessary to address underlying causes of the gait disorder.
There are different types of contractures, including:
1. Scar contracture: This type of contracture occurs when a scar tissue forms and tightens, causing a loss of movement in the affected area.
2. Neurogenic contracture: This type of contracture is caused by nerve damage and can occur after an injury or surgery.
3. Post-burn contracture: This type of contracture occurs after a burn injury and is caused by scarring and tightening of the skin and underlying tissues.
4. Congenital contracture: This type of contracture is present at birth and can be caused by genetic or environmental factors.
Signs and symptoms of contractures may include:
1. Limited range of motion
2. Pain or stiffness in the affected area
3. Skin tightening or shrinkage
4. Deformity of the affected area
Treatment options for contractures depend on the severity and cause of the condition, and may include:
1. Physical therapy to improve range of motion and strength
2. Bracing to support the affected area and prevent further tightening
3. Surgery to release or lengthen the scar tissue or tendons
4. Injections of botulinum toxin or other medications to relax the muscle and improve range of motion.
There are several types of radiculopathy, including:
1. Cervical radiculopathy: This type affects the neck and arm region and is often caused by a herniated disk or degenerative changes in the spine.
2. Thoracic radiculopathy: This type affects the chest and abdominal regions and is often caused by a tumor or injury.
3. Lumbar radiculopathy: This type affects the lower back and leg region and is often caused by a herniated disk, spinal stenosis, or degenerative changes in the spine.
4. Sacral radiculopathy: This type affects the pelvis and legs and is often caused by a tumor or injury.
The symptoms of radiculopathy can vary depending on the location and severity of the nerve compression. They may include:
1. Pain in the affected area, which can be sharp or dull and may be accompanied by numbness, tingling, or weakness.
2. Numbness or tingling sensations in the skin of the affected limb.
3. Weakness in the affected muscles, which can make it difficult to move the affected limb or perform certain activities.
4. Difficulty with coordination and balance.
5. Tremors or spasms in the affected muscles.
6. Decreased reflexes in the affected area.
7. Difficulty with bladder or bowel control (in severe cases).
Treatment for radiculopathy depends on the underlying cause and severity of the condition. Conservative treatments such as physical therapy, medication, and lifestyle changes may be effective in managing symptoms and improving function. In some cases, surgery may be necessary to relieve pressure on the nerve root.
It's important to seek medical attention if you experience any of the symptoms of radiculopathy, as early diagnosis and treatment can help prevent long-term damage and improve outcomes.
The definition of constipation varies depending on the source, but it is generally defined as having fewer than three bowel movements per week, or as experiencing difficulty passing stools for more than half of the time during a two-week period. In addition, some people may experience "functional constipation," which means that they have normal bowel habits but still experience symptoms such as bloating and discomfort.
There are several factors that can contribute to constipation, including:
* Poor diet and dehydration: A diet low in fiber and high in processed foods can lead to constipation, as can not drinking enough water.
* Lack of physical activity: Sedentary lifestyles can contribute to constipation by slowing down the digestive process.
* Medical conditions: Certain medical conditions, such as irritable bowel syndrome (IBS), thyroid disorders, and diabetes, can increase the risk of constipation.
* Medications: Some medications, such as painkillers and antidepressants, can cause constipation as a side effect.
* Hormonal changes: Changes in hormone levels during pregnancy, menopause, or other life events can lead to constipation.
Treatment for constipation depends on the underlying cause and may include dietary changes, lifestyle modifications, and medication. In severe cases, surgery may be necessary. It is important to seek medical advice if symptoms persist or worsen over time, as untreated constipation can lead to complications such as bowel obstruction, hemorrhoids, and fecal incontinence.
Some common types of movement disorders include:
1. Parkinson's disease: A degenerative disorder characterized by tremors, rigidity, bradykinesia, and postural instability.
2. Dystonia: A movement disorder characterized by sustained or intermittent muscle contractions that cause abnormal postures or movements.
3. Huntington's disease: An inherited disorder that causes progressive damage to the brain, leading to involuntary movements, cognitive decline, and psychiatric symptoms.
4. Tourette syndrome: A neurodevelopmental disorder characterized by repetitive, involuntary movements and vocalizations (tics).
5. Restless leg syndrome: A condition characterized by an uncomfortable sensation in the legs, often described as a creeping or crawling feeling, which is relieved by movement.
6. Chorea: A movement disorder characterized by rapid, jerky movements that can be triggered by emotional stress or other factors.
7. Ballism: Excessive, large, and often circular movements of the limbs, often seen in conditions such as Huntington's disease or drug-induced movements.
8. Athetosis: A slow, writhing movement that can be seen in conditions such as cerebral palsy or tardive dyskinesia.
9. Myoclonus: Sudden, brief muscle jerks or twitches that can be caused by a variety of factors, including genetic disorders, infections, and certain medications.
10. Hyperkinesis: An excessive amount of movement, often seen in conditions such as attention deficit hyperactivity disorder (ADHD) or hyperthyroidism.
Movement disorders can significantly impact an individual's quality of life, and treatment options vary depending on the specific condition and its underlying cause. Some movement disorders may be managed with medication, while others may require surgery or other interventions.
1. Essential tremor: This is the most common type of tremor, and it is characterized by a rhythmic shaking of the hands, arms, legs, or head. It can be inherited and can worsen over time.
2. Parkinson's disease: A neurodegenerative disorder that affects movement, including tremors, rigidity, and difficulty with walking.
3. Dystonia: A movement disorder that causes involuntary muscle contractions and spasms, which can result in tremors.
4. Huntington's disease: A rare genetic disorder that causes progressive damage to the brain, leading to involuntary movements, including tremors.
5. Medication-induced tremors: Certain medications, such as those used to treat psychosis, can cause tremors as a side effect.
6. Alcohol or drug withdrawal: Stopping the use of certain substances can cause tremors as part of the withdrawal process.
7. Metabolic disorders: Conditions such as hypoglycemia (low blood sugar) or hyperthyroidism (too much thyroid hormone) can cause tremors.
8. Trauma: A head injury or other trauma can sometimes cause tremors.
Tremors can be diagnosed through a physical examination and medical history, as well as through imaging tests such as CT or MRI scans. Treatment for tremors depends on the underlying cause, but may include medications, lifestyle changes, or surgery. In some cases, tremors can be managed with techniques such as physical therapy, relaxation exercises, or deep brain stimulation.
Examples of syndromes include:
1. Down syndrome: A genetic disorder caused by an extra copy of chromosome 21 that affects intellectual and physical development.
2. Turner syndrome: A genetic disorder caused by a missing or partially deleted X chromosome that affects physical growth and development in females.
3. Marfan syndrome: A genetic disorder affecting the body's connective tissue, causing tall stature, long limbs, and cardiovascular problems.
4. Alzheimer's disease: A neurodegenerative disorder characterized by memory loss, confusion, and changes in personality and behavior.
5. Parkinson's disease: A neurological disorder characterized by tremors, rigidity, and difficulty with movement.
6. Klinefelter syndrome: A genetic disorder caused by an extra X chromosome in males, leading to infertility and other physical characteristics.
7. Williams syndrome: A rare genetic disorder caused by a deletion of genetic material on chromosome 7, characterized by cardiovascular problems, developmental delays, and a distinctive facial appearance.
8. Fragile X syndrome: The most common form of inherited intellectual disability, caused by an expansion of a specific gene on the X chromosome.
9. Prader-Willi syndrome: A genetic disorder caused by a defect in the hypothalamus, leading to problems with appetite regulation and obesity.
10. Sjogren's syndrome: An autoimmune disorder that affects the glands that produce tears and saliva, causing dry eyes and mouth.
Syndromes can be diagnosed through a combination of physical examination, medical history, laboratory tests, and imaging studies. Treatment for a syndrome depends on the underlying cause and the specific symptoms and signs presented by the patient.
1. Polymyositis: This is an inflammatory disease that affects the muscles and can cause muscle weakness, pain, and stiffness.
2. Dercum's disease: This is a rare condition that causes fatty degeneration of the muscles, leading to muscle pain, weakness, and wasting.
3. Inflammatory myopathy: This is a group of conditions that cause inflammation in the muscles, leading to muscle weakness and pain.
4. Dermatomyositis: This is an inflammatory condition that affects both the skin and the muscles, causing skin rashes and muscle weakness.
5. Juvenile myositis: This is a rare condition that affects children and can cause muscle weakness, pain, and stiffness.
The symptoms of myositis can vary depending on the type of condition and its severity. Common symptoms include muscle weakness, muscle pain, stiffness, and fatigue. Other symptoms may include skin rashes, fever, and joint pain.
The diagnosis of myositis typically involves a combination of physical examination, medical history, and laboratory tests such as blood tests and muscle biopsies. Treatment for myositis depends on the underlying cause and may include medications such as corticosteroids, immunosuppressive drugs, and physical therapy. In some cases, surgery may be necessary to remove affected muscle tissue.
Causes:
1. Brain injury during fetal development or birth
2. Hypoxia (oxygen deficiency) to the brain, often due to complications during labor and delivery
3. Infections such as meningitis or encephalitis
4. Stroke or bleeding in the brain
5. Traumatic head injury
6. Genetic disorders
7. Premature birth
8. Low birth weight
9. Multiples (twins, triplets)
10. Maternal infections during pregnancy.
Symptoms:
1. Weakness or paralysis of muscles on one side of the body
2. Lack of coordination and balance
3. Difficulty with movement, posture, and gait
4. Spasticity (stiffness) or hypotonia (looseness) of muscles
5. Intellectual disability or learning disabilities
6. Seizures
7. Vision, hearing, or speech problems
8. Swallowing difficulties
9. Increased risk of infections and bone fractures
10. Delays in reaching developmental milestones.
Diagnosis:
1. Physical examination and medical history
2. Imaging tests, such as CT or MRI scans
3. Electromyography (EMG) to test muscle activity
4. Developmental assessments to evaluate cognitive and motor skills
5. Genetic testing to identify underlying causes.
Treatment:
1. Physical therapy to improve movement, balance, and strength
2. Occupational therapy to develop daily living skills and fine motor activities
3. Speech therapy for communication and swallowing difficulties
4. Medications to control seizures, spasticity, or pain
5. Surgery to correct anatomical abnormalities or release contracted muscles
6. Assistive devices, such as braces, walkers, or wheelchairs, to aid mobility and independence.
It's important to note that each individual with Cerebral Palsy may have a unique combination of symptoms and require a personalized treatment plan. With appropriate medical care and support, many individuals with Cerebral Palsy can lead fulfilling lives and achieve their goals despite the challenges they face.
The causes of LBP can be broadly classified into two categories:
1. Mechanical causes: These include strains, sprains, and injuries to the soft tissues (such as muscles, ligaments, and tendons) or bones in the lower back.
2. Non-mechanical causes: These include medical conditions such as herniated discs, degenerative disc disease, and spinal stenosis.
The symptoms of LBP can vary depending on the underlying cause and severity of the condition. Common symptoms include:
* Pain that may be localized to one side or both sides of the lower back
* Muscle spasms or stiffness
* Limited range of motion in the lower back
* Difficulty bending, lifting, or twisting
* Sciatica (pain that radiates down the legs)
* Weakness or numbness in the legs
The diagnosis of LBP is based on a combination of medical history, physical examination, and diagnostic tests such as X-rays, CT scans, or MRI.
Treatment for LBP depends on the underlying cause and severity of the condition, but may include:
* Medications such as pain relievers, muscle relaxants, or anti-inflammatory drugs
* Physical therapy to improve strength and flexibility in the lower back
* Chiropractic care to realign the spine and relieve pressure on the joints and muscles
* Injections of corticosteroids or hyaluronic acid to reduce inflammation and relieve pain
* Surgery may be considered for severe or chronic cases that do not respond to other treatments.
Prevention strategies for LBP include:
* Maintaining a healthy weight to reduce strain on the lower back
* Engaging in regular exercise to improve muscle strength and flexibility
* Using proper lifting techniques to avoid straining the lower back
* Taking regular breaks to stretch and move around if you have a job that involves sitting or standing for long periods
* Managing stress through relaxation techniques such as meditation or deep breathing.
There are several types of muscular atrophy, including:
1. Disuse atrophy: This type of atrophy occurs when a muscle is not used for a long period, leading to its degeneration.
2. Neurogenic atrophy: This type of atrophy occurs due to damage to the nerves that control muscles.
3. Dystrophic atrophy: This type of atrophy occurs due to inherited genetic disorders that affect muscle fibers.
4. Atrophy due to aging: As people age, their muscles can degenerate and lose mass and strength.
5. Atrophy due to disease: Certain diseases such as cancer, HIV/AIDS, and muscular dystrophy can cause muscular atrophy.
6. Atrophy due to infection: Infections such as polio and tetanus can cause muscular atrophy.
7. Atrophy due to trauma: Traumatic injuries can cause muscular atrophy, especially if the injury is severe and leads to prolonged immobilization.
Muscular atrophy can lead to a range of symptoms depending on the type and severity of the condition. Some common symptoms include muscle weakness, loss of motor function, muscle wasting, and difficulty performing everyday activities. Treatment for muscular atrophy depends on the underlying cause and may include physical therapy, medication, and lifestyle changes such as exercise and dietary modifications. In severe cases, surgery may be necessary to restore muscle function.
There are several different types of pain, including:
1. Acute pain: This type of pain is sudden and severe, and it usually lasts for a short period of time. It can be caused by injuries, surgery, or other forms of tissue damage.
2. Chronic pain: This type of pain persists over a long period of time, often lasting more than 3 months. It can be caused by conditions such as arthritis, fibromyalgia, or nerve damage.
3. Neuropathic pain: This type of pain results from damage to the nervous system, and it can be characterized by burning, shooting, or stabbing sensations.
4. Visceral pain: This type of pain originates in the internal organs, and it can be difficult to localize.
5. Psychogenic pain: This type of pain is caused by psychological factors such as stress, anxiety, or depression.
The medical field uses a range of methods to assess and manage pain, including:
1. Pain rating scales: These are numerical scales that patients use to rate the intensity of their pain.
2. Pain diaries: These are records that patients keep to track their pain over time.
3. Clinical interviews: Healthcare providers use these to gather information about the patient's pain experience and other relevant symptoms.
4. Physical examination: This can help healthcare providers identify any underlying causes of pain, such as injuries or inflammation.
5. Imaging studies: These can be used to visualize the body and identify any structural abnormalities that may be contributing to the patient's pain.
6. Medications: There are a wide range of medications available to treat pain, including analgesics, nonsteroidal anti-inflammatory drugs (NSAIDs), and muscle relaxants.
7. Alternative therapies: These can include acupuncture, massage, and physical therapy.
8. Interventional procedures: These are minimally invasive procedures that can be used to treat pain, such as nerve blocks and spinal cord stimulation.
It is important for healthcare providers to approach pain management with a multi-modal approach, using a combination of these methods to address the physical, emotional, and social aspects of pain. By doing so, they can help improve the patient's quality of life and reduce their suffering.
There are several theories about what might cause fibromyalgia, including:
1. Overactive nerve endings: Some research suggests that people with fibromyalgia may have overactive nerve endings that amplify pain signals.
2. Hormonal imbalance: Hormones such as cortisol and serotonin play a role in regulating pain and mood, and some studies suggest that hormonal imbalances might contribute to fibromyalgia.
3. Infections: Some research suggests that fibromyalgia may be triggered by a viral or bacterial infection, although more research is needed to confirm this theory.
4. Genetics: Fibromyalgia tends to run in families, which suggests that there may be a genetic component to the condition.
5. Environmental factors: Trauma, stress, and other environmental factors may also play a role in the development of fibromyalgia.
There is no single test for diagnosing fibromyalgia, and doctors must use a combination of physical examination, medical history, and other tests to rule out other conditions that might cause similar symptoms. Treatment for fibromyalgia typically involves a multidisciplinary approach, including medication, physical therapy, and lifestyle changes such as exercise and stress management.
Some common symptoms of fibromyalgia include:
* Widespread muscle pain and stiffness
* Fatigue and decreased energy
* Tender points on the body (areas that are painful to the touch)
* Brain fog and cognitive difficulties (such as memory loss and difficulty concentrating)
* Sleep disturbances (including insomnia and restless sleep)
* Headaches and migraines
* Digestive problems (such as irritable bowel syndrome)
* Numbness or tingling in the hands and feet
* Depression and anxiety
There is no cure for fibromyalgia, but treatment can help manage symptoms and improve quality of life. Some common medications used to treat fibromyalgia include:
* Pain relievers (such as acetaminophen or nonsteroidal anti-inflammatory drugs)
* Anti-seizure medications (which can help reduce pain and improve sleep)
* Antidepressants (which can help with mood issues and improve sleep)
* Muscle relaxants (which can help reduce muscle spasms and stiffness)
In addition to medication, physical therapy and lifestyle changes can also be helpful in managing fibromyalgia symptoms. These might include:
* Exercise programs that are tailored to the individual's needs and abilities
* Stress management techniques (such as meditation or yoga)
* Healthy sleep habits (such as establishing a consistent bedtime routine and avoiding caffeine and electronics before bedtime)
* A balanced diet and adequate hydration
* Massage therapy or other forms of relaxation techniques.
It's important to note that each person with fibromyalgia may respond differently to different treatments, so it may take some trial and error to find the right combination of medications and lifestyle changes that work best for an individual case. It's also important to work closely with a healthcare provider to monitor progress and adjust treatment plans as needed.
Electromyography
Facial electromyography
Systemic vasculitis
Bogart-Bacall syndrome
Signs and symptoms of Graves' disease
Neuromechanics
Mohamed Nasser Kotby
Somatics
Signs and symptoms of pregnancy
Tensor tympani muscle
Megavitamin-B6 syndrome
Musculocutaneous nerve
Spinal manipulation
Muscle architecture
Fall prevention
Physical therapy
Active sitting
Deltoid muscle
Pull-up (exercise)
Mirror neuron
Neural control of limb stiffness
Inflammatory myopathy
Whole-body vibration
Human leg
Bone exercise monitor
Muscle contraction
Nemaline myopathy
Injury
Incremental exercise
Lordosis
Electromyography and Nerve Conduction Studies: Background, Indications, Contraindications
Electromyography and Nerve Conduction Studies: Background, Indications, Contraindications
Electromyography: MedlinePlus Medical Encyclopedia
British Library EThOS: Simulation and analysis in electromyography
Frontiers | High Density Surface Electromyography Activity of the Lumbar Erector Spinae Muscles and Comfort/Discomfort...
The comparsion of peak vertical ground reaction forces and leg muscles electromyography during single leg drop landing between...
Legs / Feet - Thigh - Incapable of Moving Leg - Electromyography EMG Neurology | Medical Procedures | MediBid
Electromyography
Electromyography (EMG) Activity for Back Training
Electromyography (EMG) and Nerve Conduction Studies | NeuropathyCommons
QEMG - Quantitative Electromyography and Time Series Analysis (QEMG)
Reviewer, Journal of Electromyography and Kinesiology (External organization) - Central Michigan University
I need a help With my Electromyography lab report - Answers Market
A comparison of surface acquired uterine electromyography and intrauterine pressure catheter to assess uterine activity. | Am...
Could Relative Movement Between the Adductor Muscles and the Skin Invalidate Surface Electromyography Measurement? | J Appl...
Electromyography and asymmetry index of masticatory muscles in undergraduate students with temporomandibular disorders
Electromyography (EMG) and equine lameness; Expanding clinical research into equine biomechanics. - Delsys Europe
Technical Services for Motility Disorders | Children's Hospital of Philadelphia
How to Code for Electromyography (EMG) and Nerve Conduction Studies (NCS)? | HELPDESKDIRECT CONSULTANCY SERVICE
Evidence of Emotion-Antecedent Appraisal Checks in Electroencephalography and Facial Electromyography - The University of...
MappEMG: Supporting Musical Expression with Vibrotactile Feedback by Capturing Gestural Features through Electromyography -...
Surface Electromyography Meets Biomechanics or Bringing sEMG t... - RWTH AACHEN UNIVERSITY Institut für Angewandte...
BodyWell Group in Canterbury • Read 131 Reviews
A neural network that finds a naturalistic solution for the production of muscle activity
Type V Glycogen Storage Disease Workup: Laboratory Studies, Other Tests, Procedures
Table 2 - Nodding Syndrome - Volume 19, Number 9-September 2013 - Emerging Infectious Diseases journal - CDC
Transgender man in Ecuador makes history with pregnancy - WINK News
Abstract] Determining Feature Relevance for the Grouping of Motor Unit Action Potentials through Generative Topographic Mapping
Proceedings of the first workshop on peripheral machine interfaces: Going beyond traditional surface electromyography<...
Surface12
- A comparison of surface acquired uterine electromyography and intrauterine pressure catheter to assess uterine activity. (bvsalud.org)
- Could Relative Movement Between the Adductor Muscles and the Skin Invalidate Surface Electromyography Measurement? (bvsalud.org)
- Therefore, relative movement between the overlying skin and the muscle belly could lead to a shift in the position of surface electromyography (EMG) electrodes and contamination of EMG signals with activity from neighboring muscles . (bvsalud.org)
- Methods: Surface electromyography (EMG) of the right and left masseter and temporalis muscles was performed in 126 undergraduate students at rest and at maximal voluntary contraction. (bvsalud.org)
- Through novel use of surface electromyography (sEMG), this research will be the first comprehensive sensor-based quantification of muscle function during both normal equine locomotion and during lameness. (delsyseurope.com)
- Although gait analysis technologies have been embraced by veterinarians in both practice and research to quantify movement asymmetries that occur during lameness, the clinical application of surface electromyography for evaluating equine locomotion is in its infancy. (delsyseurope.com)
- This demo paper presents a work-in-progress implementation of an integrated set-up, the MappEMG, to produce a vibrotactile feedback based on surface electromyography data of pianists' abdominal muscle activations. (hal.science)
- Surface Electromyography Meets Biomechanics or Bringing sEMG t. (rwth-aachen.de)
- A wireless electromyography system with 9 pairs of bipolar surface electrodes used to record the electromyography activity of back and lower limb muscles (sample rate: 2000 Hz). (ac.ir)
- To assess the diagnostic accuracy of the surface electromyography (sEMG) parameters associated with referred anterior knee pain in diagnosing patellofemoral pain syndrome (PFPS). (qxmd.com)
- Surface electromyography is a method able to detect subtle changes in muscle activity. (cdc.gov)
- The study on the relationship of associated reactions with spasticity was made on the 20 hemiplegic subjects in terms of electrical muscle activity and elbow and ankle movement in the paretic arm and leg using surface electromyography and clinical goniometry respectively. (who.int)
Muscles6
- Electromyography (EMG) is a test that checks the health of the muscles and the nerves that control the muscles. (medlineplus.gov)
- An Electromyography (EMG) is conducted to analyze and record electrical activity of the muscles. (medibid.com)
- Electromyography (EMG) is a technique for evaluating and recording physiologic properties of muscles at rest and while contracting. (chemeurope.com)
- Electromyography and nerve conduction studies, commonly known as "EMG," are diagnostic tests that measure the electrical activities of peripheral nerves (outside the spinal cord) and muscles. (neuropathycommons.org)
- The purpose of this study was to investigate the effects of long-term lumbar brace on the electromyography activities of lower limb muscles during walking in low back pain older adalts with pronated foot. (ac.ir)
- The electrical activity of the lower limb and trunk muscles was recorded by the electromyography system during walking activity in both pre and post-test. (ac.ir)
Needle3
- Electrodiagnostic testing encompasses a range of specialized tests, including nerve conduction studies (NCS) and needle electromyography (EMG), that are used to evaluate the conduction of electrical impulses along peripheral nerves. (medscape.com)
- Electromyography (EMG) usually involves inserting a hair-thin needle into specific muscle to assess the muscle's electrical activity with and without muscle movement. (neuropathycommons.org)
- Needle gressive myopathy, but the disease spectrum electromyography showed polyphasicity, includes patients whose disease is much decreased duration and latency of motor more severe [ 8 ]. (who.int)
Electroencephalography1
- In particular, we focused on the automatic detection of five appraisal checks-novelty, intrinsic pleasantness, goal conduciveness, control, and power-in electroencephalography (EEG) and facial electromyography (EMG) signals. (liverpool.ac.uk)
Jitter1
- Single-fiber electromyography may reveal increased jitter. (medscape.com)
Nerve3
- Electromyography and nerve conduction studies are important and helpful in diagnosing motor neuron diseases (ALS), spinal root diseases (disc herniations), peripheral neuropathies (diabetes), single nerve damage (carpal tunnel syndrome), neuromuscular transmission disorders (myasthenia gravis), and primary muscle diseases (muscular dystrophies). (neuropathycommons.org)
- How to Code for Electromyography (EMG) and Nerve Conduction Studies (NCS)? (helpdeskdirect.net)
- Electrodiagnostic medicine (EDX) evaluation, which includes electromyography (EMG) and nerve conduction studies (NCS), is an important component of the clinical evaluation of patients with disorders of the peripheral and/or central nervous system. (helpdeskdirect.net)
Quantitative2
- The study of the techniques to do so are called quantitative electromyography , or QEMG. (uoguelph.ca)
- We would like to share this software with other people interested in quantitative electromyography (QEMG). (uoguelph.ca)
Electrical1
- Electrical uterine electromyography yields information about uterine contractility comparable to that obtained with IUPC. (bvsalud.org)
Muscle1
- Findings from electromyography of resting muscle are normal. (medscape.com)
Normal2
- In contrast to most GSDs, findings upon electromyography may be normal. (medscape.com)
- Electromyography was normal. (who.int)
Pain1
- Diagnostic accuracy of the electromyography parameters associated with anterior knee pain in the diagnosis of patellofemoral pain syndrome. (qxmd.com)
Surface4
- Methods: Surface electromyography (EMG) of the right and left masseter and temporalis muscles was performed in 126 undergraduate students at rest and at maximal voluntary contraction. (bvsalud.org)
- Assessment of neck and shoulder muscle fatigue using discrete wavelet transforms of surface electromyography. (cdc.gov)
- Although, in recent years, a number of researchers have used discrete wavelet transforms (DWT) of surface electromyography (SEMG) to evaluate muscle fatigue, its application to neck and shoulder muscle fatigue assessment is not well established. (cdc.gov)
- Surface electromyography was used to quantify synergy indices stabilizing center of pressure shifts in the anterior-posterior direction during a load-release task. (nih.gov)
Motor1
- Measuring the spatial dimensions of a single motor unit remains a challenging problem, and current techniques, such as scanning electromyography (EMG), tend to underestimate the true dimensions. (nih.gov)