Recording of the moment-to-moment electromotive forces of the HEART as projected onto various sites on the body's surface, delineated as a scalar function of time. The recording is monitored by a tracing on slow moving chart paper or by observing it on a cardioscope, which is a CATHODE RAY TUBE DISPLAY.
Method in which prolonged electrocardiographic recordings are made on a portable tape recorder (Holter-type system) or solid-state device ("real-time" system), while the patient undergoes normal daily activities. It is useful in the diagnosis and management of intermittent cardiac arrhythmias and transient myocardial ischemia.
Controlled physical activity which is performed in order to allow assessment of physiological functions, particularly cardiovascular and pulmonary, but also aerobic capacity. Maximal (most intense) exercise is usually required but submaximal exercise is also used.
Ultrasonic recording of the size, motion, and composition of the heart and surrounding tissues. The standard approach is transthoracic.
Any disturbances of the normal rhythmic beating of the heart or MYOCARDIAL CONTRACTION. Cardiac arrhythmias can be classified by the abnormalities in HEART RATE, disorders of electrical impulse generation, or impulse conduction.
The measurement of magnetic fields generated by electric currents from the heart. The measurement of these fields provides information which is complementary to that provided by ELECTROCARDIOGRAPHY.
Recording of the moment-to-moment electromotive forces of the heart on a plane of the body surface delineated as a vector function of time.
Computer-assisted processing of electric, ultrasonic, or electronic signals to interpret function and activity.
Pressure, burning, or numbness in the chest.
An impulse-conducting system composed of modified cardiac muscle, having the power of spontaneous rhythmicity and conduction more highly developed than the rest of the heart.
NECROSIS of the MYOCARDIUM caused by an obstruction of the blood supply to the heart (CORONARY CIRCULATION).
Enlargement of the LEFT VENTRICLE of the heart. This increase in ventricular mass is attributed to sustained abnormal pressure or volume loads and is a contributor to cardiovascular morbidity and mortality.
The number of times the HEART VENTRICLES contract per unit of time, usually per minute.
Act of listening for sounds within the heart.
The hollow, muscular organ that maintains the circulation of the blood.
In screening and diagnostic tests, the probability that a person with a positive test is a true positive (i.e., has the disease), is referred to as the predictive value of a positive test; whereas, the predictive value of a negative test is the probability that the person with a negative test does not have the disease. Predictive value is related to the sensitivity and specificity of the test.
Binary classification measures to assess test results. Sensitivity or recall rate is the proportion of true positives. Specificity is the probability of correctly determining the absence of a condition. (From Last, Dictionary of Epidemiology, 2d ed)
Radiography of the vascular system of the heart muscle after injection of a contrast medium.
An imbalance between myocardial functional requirements and the capacity of the CORONARY VESSELS to supply sufficient blood flow. It is a form of MYOCARDIAL ISCHEMIA (insufficient blood supply to the heart muscle) caused by a decreased capacity of the coronary vessels.
A phosphodiesterase inhibitor that blocks uptake and metabolism of adenosine by erythrocytes and vascular endothelial cells. Dipyridamole also potentiates the antiaggregating action of prostacyclin. (From AMA Drug Evaluations Annual, 1994, p752)
Unexpected rapid natural death due to cardiovascular collapse within one hour of initial symptoms. It is usually caused by the worsening of existing heart diseases. The sudden onset of symptoms, such as CHEST PAIN and CARDIAC ARRHYTHMIAS, particularly VENTRICULAR TACHYCARDIA, can lead to the loss of consciousness and cardiac arrest followed by biological death. (from Braunwald's Heart Disease: A Textbook of Cardiovascular Medicine, 7th ed., 2005)
A disorder of cardiac function caused by insufficient blood flow to the muscle tissue of the heart. The decreased blood flow may be due to narrowing of the coronary arteries (CORONARY ARTERY DISEASE), to obstruction by a thrombus (CORONARY THROMBOSIS), or less commonly, to diffuse narrowing of arterioles and other small vessels within the heart. Severe interruption of the blood supply to the myocardial tissue may result in necrosis of cardiac muscle (MYOCARDIAL INFARCTION).
A heavy, bluish white metal, atomic number 81, atomic weight [204.382; 204.385], symbol Tl.
Impaired conduction of cardiac impulse that can occur anywhere along the conduction pathway, such as between the SINOATRIAL NODE and the right atrium (SA block) or between atria and ventricles (AV block). Heart blocks can be classified by the duration, frequency, or completeness of conduction block. Reversibility depends on the degree of structural or functional defects.
Pathological conditions involving the HEART including its structural and functional abnormalities.
Monitoring of FETAL HEART frequency before birth in order to assess impending prematurity in relation to the pattern or intensity of antepartum UTERINE CONTRACTION.
Observation of a population for a sufficient number of persons over a sufficient number of years to generate incidence or mortality rates subsequent to the selection of the study group.
The period shortly before, during, and immediately after giving birth.
The graphic recording of chest wall movement due to cardiac impulses.
A catecholamine derivative with specificity for BETA-1 ADRENERGIC RECEPTORS. It is commonly used as a cardiotonic agent after CARDIAC SURGERY and during DOBUTAMINE STRESS ECHOCARDIOGRAPHY.
A group of diseases in which the dominant feature is the involvement of the CARDIAC MUSCLE itself. Cardiomyopathies are classified according to their predominant pathophysiological features (DILATED CARDIOMYOPATHY; HYPERTROPHIC CARDIOMYOPATHY; RESTRICTIVE CARDIOMYOPATHY) or their etiological/pathological factors (CARDIOMYOPATHY, ALCOHOLIC; ENDOCARDIAL FIBROELASTOSIS).
Simple rapid heartbeats caused by rapid discharge of impulses from the SINOATRIAL NODE, usually between 100 and 180 beats/min in adults. It is characterized by a gradual onset and termination. Sinus tachycardia is common in infants, young children, and adults during strenuous physical activities.
Transmission of the readings of instruments to a remote location by means of wires, radio waves, or other means. (McGraw-Hill Dictionary of Scientific and Technical Terms, 4th ed)
The study of the heart, its physiology, and its functions.
The continuous measurement of physiological processes, blood pressure, heart rate, renal output, reflexes, respiration, etc., in a patient or experimental animal; includes pharmacologic monitoring, the measurement of administered drugs or their metabolites in the blood, tissues, or urine.
Abnormal cardiac rhythm that is characterized by rapid, uncoordinated firing of electrical impulses in the upper chambers of the heart (HEART ATRIA). In such case, blood cannot be effectively pumped into the lower chambers of the heart (HEART VENTRICLES). It is caused by abnormal impulse generation.
A transient loss of consciousness and postural tone caused by diminished blood flow to the brain (i.e., BRAIN ISCHEMIA). Presyncope refers to the sensation of lightheadedness and loss of strength that precedes a syncopal event or accompanies an incomplete syncope. (From Adams et al., Principles of Neurology, 6th ed, pp367-9)
A type of cardiac arrhythmia with premature contractions of the HEART VENTRICLES. It is characterized by the premature QRS complex on ECG that is of abnormal shape and great duration (generally >129 msec). It is the most common form of all cardiac arrhythmias. Premature ventricular complexes have no clinical significance except in concurrence with heart diseases.
The symptom of paroxysmal pain consequent to MYOCARDIAL ISCHEMIA usually of distinctive character, location and radiation. It is thought to be provoked by a transient stressful situation during which the oxygen requirements of the MYOCARDIUM exceed that supplied by the CORONARY CIRCULATION.
Imaging of a ventricle of the heart after the injection of a radioactive contrast medium. The technique is less invasive than cardiac catheterization and is used to assess ventricular function.
A form of ventricular pre-excitation characterized by a short PR interval and a long QRS interval with a delta wave. In this syndrome, atrial impulses are abnormally conducted to the HEART VENTRICLES via an ACCESSORY CONDUCTING PATHWAY that is located between the wall of the right or left atria and the ventricles, also known as a BUNDLE OF KENT. The inherited form can be caused by mutation of PRKAG2 gene encoding a gamma-2 regulatory subunit of AMP-activated protein kinase.
A prediction of the probable outcome of a disease based on a individual's condition and the usual course of the disease as seen in similar situations.
An abnormally rapid ventricular rhythm usually in excess of 150 beats per minute. It is generated within the ventricle below the BUNDLE OF HIS, either as autonomic impulse formation or reentrant impulse conduction. Depending on the etiology, onset of ventricular tachycardia can be paroxysmal (sudden) or nonparoxysmal, its wide QRS complexes can be uniform or polymorphic, and the ventricular beating may be independent of the atrial beating (AV dissociation).
Cardiac arrhythmias that are characterized by excessively slow HEART RATE, usually below 50 beats per minute in human adults. They can be classified broadly into SINOATRIAL NODE dysfunction and ATRIOVENTRICULAR BLOCK.
A generic expression for any tachycardia that originates above the BUNDLE OF HIS.
The hospital department responsible for the administration and provision of diagnostic and therapeutic services for the cardiac patient.
Paramedical personnel trained to provide basic emergency care and life support under the supervision of physicians and/or nurses. These services may be carried out at the site of the emergency, in the ambulance, or in a health care institution.
A form of heart block in which the electrical stimulation of HEART VENTRICLES is interrupted at either one of the branches of BUNDLE OF HIS thus preventing the simultaneous depolarization of the two ventricles.
Studies in which individuals or populations are followed to assess the outcome of exposures, procedures, or effects of a characteristic, e.g., occurrence of disease.
One of the three polypeptide chains that make up the TROPONIN complex. It is a cardiac-specific protein that binds to TROPOMYOSIN. It is released from damaged or injured heart muscle cells (MYOCYTES, CARDIAC). Defects in the gene encoding troponin T result in FAMILIAL HYPERTROPHIC CARDIOMYOPATHY.
A form of CARDIAC MUSCLE disease, characterized by left and/or right ventricular hypertrophy (HYPERTROPHY, LEFT VENTRICULAR; HYPERTROPHY, RIGHT VENTRICULAR), frequent asymmetrical involvement of the HEART SEPTUM, and normal or reduced left ventricular volume. Risk factors include HYPERTENSION; AORTIC STENOSIS; and gene MUTATION; (FAMILIAL HYPERTROPHIC CARDIOMYOPATHY).
Unstable isotopes of thallium that decay or disintegrate emitting radiation. Tl atoms with atomic weights 198-202, 204, and 206-210 are thallium radioisotopes.
A condition in which the LEFT VENTRICLE of the heart was functionally impaired. This condition usually leads to HEART FAILURE; MYOCARDIAL INFARCTION; and other cardiovascular complications. Diagnosis is made by measuring the diminished ejection fraction and a depressed level of motility of the left ventricular wall.
Congenital, inherited, or acquired anomalies of the CARDIOVASCULAR SYSTEM, including the HEART and BLOOD VESSELS.
The lower right and left chambers of the heart. The right ventricle pumps venous BLOOD into the LUNGS and the left ventricle pumps oxygenated blood into the systemic arterial circulation.
Elements of limited time intervals, contributing to particular results or situations.
Impaired impulse conduction from HEART ATRIA to HEART VENTRICLES. AV block can mean delayed or completely blocked impulse conduction.
Systematic and thorough inspection of the patient for physical signs of disease or abnormality.
Examinations used to diagnose and treat heart conditions.
Pathological processes of CORONARY ARTERIES that may derive from a congenital abnormality, atherosclerotic, or non-atherosclerotic cause.
An aspect of personal behavior or lifestyle, environmental exposure, or inborn or inherited characteristic, which, on the basis of epidemiologic evidence, is known to be associated with a health-related condition considered important to prevent.
Electric conductors through which electric currents enter or leave a medium, whether it be an electrolytic solution, solid, molten mass, gas, or vacuum.
Inflammation of the PERICARDIUM from various origins, such as infection, neoplasm, autoimmune process, injuries, or drug-induced. Pericarditis usually leads to PERICARDIAL EFFUSION, or CONSTRICTIVE PERICARDITIS.
The hemodynamic and electrophysiological action of the left HEART VENTRICLE. Its measurement is an important aspect of the clinical evaluation of patients with heart disease to determine the effects of the disease on cardiac performance.
Heart sounds caused by vibrations resulting from the flow of blood through the heart. Heart murmurs can be examined by HEART AUSCULTATION, and analyzed by their intensity (6 grades), duration, timing (systolic, diastolic, or continuous), location, transmission, and quality (musical, vibratory, blowing, etc).
The ENTERIC NERVOUS SYSTEM; PARASYMPATHETIC NERVOUS SYSTEM; and SYMPATHETIC NERVOUS SYSTEM taken together. Generally speaking, the autonomic nervous system regulates the internal environment during both peaceful activity and physical or emotional stress. Autonomic activity is controlled and integrated by the CENTRAL NERVOUS SYSTEM, especially the HYPOTHALAMUS and the SOLITARY NUCLEUS, which receive information relayed from VISCERAL AFFERENTS.
Graphic registration of the heart sounds picked up as vibrations and transformed by a piezoelectric crystal microphone into a varying electrical output according to the stresses imposed by the sound waves. The electrical output is amplified by a stethograph amplifier and recorded by a device incorporated into the electrocardiograph or by a multichannel recording machine.
Abnormally rapid heartbeat, usually with a HEART RATE above 100 beats per minute for adults. Tachycardia accompanied by disturbance in the cardiac depolarization (cardiac arrhythmia) is called tachyarrhythmia.
X-ray visualization of the chest and organs of the thoracic cavity. It is not restricted to visualization of the lungs.
Recording of regional electrophysiological information by analysis of surface potentials to give a complete picture of the effects of the currents from the heart on the body surface. It has been applied to the diagnosis of old inferior myocardial infarction, localization of the bypass pathway in Wolff-Parkinson-White syndrome, recognition of ventricular hypertrophy, estimation of the size of a myocardial infarct, and the effects of different interventions designed to reduce infarct size. The limiting factor at present is the complexity of the recording and analysis, which requires 100 or more electrodes, sophisticated instrumentation, and dedicated personnel. (Braunwald, Heart Disease, 4th ed)
Measurement of intracardiac blood flow using an M-mode and/or two-dimensional (2-D) echocardiogram while simultaneously recording the spectrum of the audible Doppler signal (e.g., velocity, direction, amplitude, intensity, timing) reflected from the moving column of red blood cells.
The abrupt cessation of all vital bodily functions, manifested by the permanent loss of total cerebral, respiratory, and cardiovascular functions.
A method of recording heart motion and internal structures by combining ultrasonic imaging with exercise testing (EXERCISE TEST) or pharmacologic stress.
Complications that affect patients during surgery. They may or may not be associated with the disease for which the surgery is done, or within the same surgical procedure.
A form of CARDIAC MUSCLE disease that is characterized by ventricular dilation, VENTRICULAR DYSFUNCTION, and HEART FAILURE. Risk factors include SMOKING; ALCOHOL DRINKING; HYPERTENSION; INFECTION; PREGNANCY; and mutations in the LMNA gene encoding LAMIN TYPE A, a NUCLEAR LAMINA protein.
A transient left ventricular apical dysfunction or ballooning accompanied by electrocardiographic (ECG) T wave inversions. This abnormality is associated with high levels of CATECHOLAMINES, either administered or endogenously secreted from a tumor or during extreme stress.
An autosomal dominant defect of cardiac conduction that is characterized by an abnormal ST-segment in leads V1-V3 on the ELECTROCARDIOGRAM resembling a right BUNDLE-BRANCH BLOCK; high risk of VENTRICULAR TACHYCARDIA; or VENTRICULAR FIBRILLATION; SYNCOPAL EPISODE; and possible sudden death. This syndrome is linked to mutations of gene encoding the cardiac SODIUM CHANNEL alpha subunit.
Expenditure of energy during PHYSICAL ACTIVITY. Intensity of exertion may be measured by rate of OXYGEN CONSUMPTION; HEAT produced, or HEART RATE. Perceived exertion, a psychological measure of exertion, is included.
A condition that is characterized by episodes of fainting (SYNCOPE) and varying degree of ventricular arrhythmia as indicated by the prolonged QT interval. The inherited forms are caused by mutation of genes encoding cardiac ion channel proteins. The two major forms are ROMANO-WARD SYNDROME and JERVELL-LANGE NIELSEN SYNDROME.
The amount of BLOOD pumped out of the HEART per beat, not to be confused with cardiac output (volume/time). It is calculated as the difference between the end-diastolic volume and the end-systolic volume.
Radiography of the heart and great vessels after injection of a contrast medium.
Persistently high systemic arterial BLOOD PRESSURE. Based on multiple readings (BLOOD PRESSURE DETERMINATION), hypertension is currently defined as when SYSTOLIC PRESSURE is consistently greater than 140 mm Hg or when DIASTOLIC PRESSURE is consistently 90 mm Hg or more.
Activities or games, usually involving physical effort or skill. Reasons for engagement in sports include pleasure, competition, and/or financial reward.
The total number of cases of a given disease in a specified population at a designated time. It is differentiated from INCIDENCE, which refers to the number of new cases in the population at a given time.
Application of computer programs designed to assist the physician in solving a diagnostic problem.
Evaluation undertaken to assess the results or consequences of management and procedures used in combating disease in order to determine the efficacy, effectiveness, safety, and practicability of these interventions in individual cases or series.
Agents that have a strengthening effect on the heart or that can increase cardiac output. They may be CARDIAC GLYCOSIDES; SYMPATHOMIMETICS; or other drugs. They are used after MYOCARDIAL INFARCT; CARDIAC SURGICAL PROCEDURES; in SHOCK; or in congestive heart failure (HEART FAILURE).
Agents used for the treatment or prevention of cardiac arrhythmias. They may affect the polarization-repolarization phase of the action potential, its excitability or refractoriness, or impulse conduction or membrane responsiveness within cardiac fibers. Anti-arrhythmia agents are often classed into four main groups according to their mechanism of action: sodium channel blockade, beta-adrenergic blockade, repolarization prolongation, or calcium channel blockade.
The chambers of the heart, to which the BLOOD returns from the circulation.
The qualitative or quantitative estimation of the likelihood of adverse effects that may result from exposure to specified health hazards or from the absence of beneficial influences. (Last, Dictionary of Epidemiology, 1988)
A method of computed tomography that uses radionuclides which emit a single photon of a given energy. The camera is rotated 180 or 360 degrees around the patient to capture images at multiple positions along the arc. The computer is then used to reconstruct the transaxial, sagittal, and coronal images from the 3-dimensional distribution of radionuclides in the organ. The advantages of SPECT are that it can be used to observe biochemical and physiological processes as well as size and volume of the organ. The disadvantage is that, unlike positron-emission tomography where the positron-electron annihilation results in the emission of 2 photons at 180 degrees from each other, SPECT requires physical collimation to line up the photons, which results in the loss of many available photons and hence degrades the image.
A transferase that catalyzes formation of PHOSPHOCREATINE from ATP + CREATINE. The reaction stores ATP energy as phosphocreatine. Three cytoplasmic ISOENZYMES have been identified in human tissues: the MM type from SKELETAL MUSCLE, the MB type from myocardial tissue and the BB type from nervous tissue as well as a mitochondrial isoenzyme. Macro-creatine kinase refers to creatine kinase complexed with other serum proteins.
A small nodular mass of specialized muscle fibers located in the interatrial septum near the opening of the coronary sinus. It gives rise to the atrioventricular bundle of the conduction system of the heart.
A device designed to stimulate, by electric impulses, contraction of the heart muscles. It may be temporary (external) or permanent (internal or internal-external).
Regulation of the rate of contraction of the heart muscles by an artificial pacemaker.
A heterogeneous condition in which the heart is unable to pump out sufficient blood to meet the metabolic need of the body. Heart failure can be caused by structural defects, functional abnormalities (VENTRICULAR DYSFUNCTION), or a sudden overload beyond its capacity. Chronic heart failure is more common than acute heart failure which results from sudden insult to cardiac function, such as MYOCARDIAL INFARCTION.
Studies used to test etiologic hypotheses in which inferences about an exposure to putative causal factors are derived from data relating to characteristics of persons under study or to events or experiences in their past. The essential feature is that some of the persons under study have the disease or outcome of interest and their characteristics are compared with those of unaffected persons.
A technetium imaging agent used to reveal blood-starved cardiac tissue during a heart attack.
Tests used in the analysis of the hemic system.
The statistical reproducibility of measurements (often in a clinical context), including the testing of instrumentation or techniques to obtain reproducible results. The concept includes reproducibility of physiological measurements, which may be used to develop rules to assess probability or prognosis, or response to a stimulus; reproducibility of occurrence of a condition; and reproducibility of experimental results.
Procedures in which placement of CARDIAC CATHETERS is performed for therapeutic or diagnostic procedures.
The muscle tissue of the HEART. It is composed of striated, involuntary muscle cells (MYOCYTES, CARDIAC) connected to form the contractile pump to generate blood flow.
A condition in which the RIGHT VENTRICLE of the heart was functionally impaired. This condition usually leads to HEART FAILURE or MYOCARDIAL INFARCTION, and other cardiovascular complications. Diagnosis is made by measuring the diminished ejection fraction and a depressed level of motility of the right ventricular wall.
PRESSURE of the BLOOD on the ARTERIES and other BLOOD VESSELS.
Types of spiral computed tomography technology in which multiple slices of data are acquired simultaneously improving the resolution over single slice acquisition technology.
Enlargement of the RIGHT VENTRICLE of the heart. This increase in ventricular mass is often attributed to PULMONARY HYPERTENSION and is a contributor to cardiovascular morbidity and mortality.
Developmental abnormalities involving structures of the heart. These defects are present at birth but may be discovered later in life.
Ultrasonic recording of the size, motion, and composition of the heart and surrounding tissues using a transducer placed in the esophagus.
Surgical therapy of ischemic coronary artery disease achieved by grafting a section of saphenous vein, internal mammary artery, or other substitute between the aorta and the obstructed coronary artery distal to the obstructive lesion.
A PEPTIDE that is secreted by the BRAIN and the HEART ATRIA, stored mainly in cardiac ventricular MYOCARDIUM. It can cause NATRIURESIS; DIURESIS; VASODILATION; and inhibits secretion of RENIN and ALDOSTERONE. It improves heart function. It contains 32 AMINO ACIDS.
Care given during the period prior to undergoing surgery when psychological and physical preparations are made according to the special needs of the individual patient. This period spans the time between admission to the hospital to the time the surgery begins. (From Dictionary of Health Services Management, 2d ed)
The creation and display of functional images showing where the blood is flowing into the MYOCARDIUM by following over time the distribution of tracers injected into the blood stream.
Studies determining the effectiveness or value of processes, personnel, and equipment, or the material on conducting such studies. For drugs and devices, CLINICAL TRIALS AS TOPIC; DRUG EVALUATION; and DRUG EVALUATION, PRECLINICAL are available.
Difficult or labored breathing.
A characteristic symptom complex.
Spasm of the large- or medium-sized coronary arteries.
Theoretical representations that simulate the behavior or activity of the cardiovascular system, processes, or phenomena; includes the use of mathematical equations, computers and other electronic equipment.
A distribution in which a variable is distributed like the sum of the squares of any given independent random variable, each of which has a normal distribution with mean of zero and variance of one. The chi-square test is a statistical test based on comparison of a test statistic to a chi-square distribution. The oldest of these tests are used to detect whether two or more population distributions differ from one another.
Inflammatory processes of the muscular walls of the heart (MYOCARDIUM) which result in injury to the cardiac muscle cells (MYOCYTES, CARDIAC). Manifestations range from subclinical to sudden death (DEATH, SUDDEN). Myocarditis in association with cardiac dysfunction is classified as inflammatory CARDIOMYOPATHY usually caused by INFECTION, autoimmune diseases, or responses to toxic substances. Myocarditis is also a common cause of DILATED CARDIOMYOPATHY and other cardiomyopathies.
One of the three polypeptide chains that make up the TROPONIN complex. It inhibits F-actin-myosin interactions.
Measurable and quantifiable biological parameters (e.g., specific enzyme concentration, specific hormone concentration, specific gene phenotype distribution in a population, presence of biological substances) which serve as indices for health- and physiology-related assessments, such as disease risk, psychiatric disorders, environmental exposure and its effects, disease diagnosis, metabolic processes, substance abuse, pregnancy, cell line development, epidemiologic studies, etc.
A class of statistical procedures for estimating the survival function (function of time, starting with a population 100% well at a given time and providing the percentage of the population still well at later times). The survival analysis is then used for making inferences about the effects of treatments, prognostic factors, exposures, and other covariates on the function.
Services specifically designed, staffed, and equipped for the emergency care of patients.
Drugs used to cause dilation of the blood vessels.
The movement and the forces involved in the movement of the blood through the CARDIOVASCULAR SYSTEM.
Levels within a diagnostic group which are established by various measurement criteria applied to the seriousness of a patient's disorder.
A set of techniques used when variation in several variables has to be studied simultaneously. In statistics, multivariate analysis is interpreted as any analytic method that allows simultaneous study of two or more dependent variables.
Pathological conditions involving any of the various HEART VALVES and the associated structures (PAPILLARY MUSCLES and CHORDAE TENDINEAE).
Contractile activity of the MYOCARDIUM.
The domestic dog, Canis familiaris, comprising about 400 breeds, of the carnivore family CANIDAE. They are worldwide in distribution and live in association with people. (Walker's Mammals of the World, 5th ed, p1065)
The range or frequency distribution of a measurement in a population (of organisms, organs or things) that has not been selected for the presence of disease or abnormality.
The hemodynamic and electrophysiological action of the right HEART VENTRICLE.
Pathologic processes that affect patients after a surgical procedure. They may or may not be related to the disease for which the surgery was done, and they may or may not be direct results of the surgery.
Narrowing or constriction of a coronary artery.
Period of contraction of the HEART, especially of the HEART VENTRICLES.
Age as a constituent element or influence contributing to the production of a result. It may be applicable to the cause or the effect of a circumstance. It is used with human or animal concepts but should be differentiated from AGING, a physiological process, and TIME FACTORS which refers only to the passage of time.
Tomography using x-ray transmission and a computer algorithm to reconstruct the image.
Pathological conditions involving the CARDIOVASCULAR SYSTEM including the HEART; the BLOOD VESSELS; or the PERICARDIUM.
Implantable devices which continuously monitor the electrical activity of the heart and automatically detect and terminate ventricular tachycardia (TACHYCARDIA, VENTRICULAR) and VENTRICULAR FIBRILLATION. They consist of an impulse generator, batteries, and electrodes.
The period following a surgical operation.
Disease having a short and relatively severe course.
Maleness or femaleness as a constituent element or influence contributing to the production of a result. It may be applicable to the cause or effect of a circumstance. It is used with human or animal concepts but should be differentiated from SEX CHARACTERISTICS, anatomical or physiological manifestations of sex, and from SEX DISTRIBUTION, the number of males and females in given circumstances.
A method of comparing the cost of a program with its expected benefits in dollars (or other currency). The benefit-to-cost ratio is a measure of total return expected per unit of money spent. This analysis generally excludes consideration of factors that are not measured ultimately in economic terms. Cost effectiveness compares alternative ways to achieve a specific set of results.
The record of descent or ancestry, particularly of a particular condition or trait, indicating individual family members, their relationships, and their status with respect to the trait or condition.
Statistical models which describe the relationship between a qualitative dependent variable (that is, one which can take only certain discrete values, such as the presence or absence of a disease) and an independent variable. A common application is in epidemiology for estimating an individual's risk (probability of a disease) as a function of a given risk factor.
The circulation of blood through the CORONARY VESSELS of the HEART.
Post-systolic relaxation of the HEART, especially the HEART VENTRICLES.
The probability that an event will occur. It encompasses a variety of measures of the probability of a generally unfavorable outcome.
Compounds that are used in medicine as sources of radiation for radiotherapy and for diagnostic purposes. They have numerous uses in research and industry. (Martindale, The Extra Pharmacopoeia, 30th ed, p1161)
Non-invasive method of demonstrating internal anatomy based on the principle that atomic nuclei in a strong magnetic field absorb pulses of radiofrequency energy and emit them as radiowaves which can be reconstructed into computerized images. The concept includes proton spin tomographic techniques.
The veins and arteries of the HEART.
A class of statistical methods applicable to a large set of probability distributions used to test for correlation, location, independence, etc. In most nonparametric statistical tests, the original scores or observations are replaced by another variable containing less information. An important class of nonparametric tests employs the ordinal properties of the data. Another class of tests uses information about whether an observation is above or below some fixed value such as the median, and a third class is based on the frequency of the occurrence of runs in the data. (From McGraw-Hill Dictionary of Scientific and Technical Terms, 4th ed, p1284; Corsini, Concise Encyclopedia of Psychology, 1987, p764-5)
Studies which start with the identification of persons with a disease of interest and a control (comparison, referent) group without the disease. The relationship of an attribute to the disease is examined by comparing diseased and non-diseased persons with regard to the frequency or levels of the attribute in each group.
The hemodynamic and electrophysiological action of the HEART VENTRICLES.
A procedure consisting of a sequence of algebraic formulas and/or logical steps to calculate or determine a given task.
Studies in which the presence or absence of disease or other health-related variables are determined in each member of the study population or in a representative sample at one particular time. This contrasts with LONGITUDINAL STUDIES which are followed over a period of time.
Studies in which subsets of a defined population are identified. These groups may or may not be exposed to factors hypothesized to influence the probability of the occurrence of a particular disease or other outcome. Cohorts are defined populations which, as a whole, are followed in an attempt to determine distinguishing subgroup characteristics.
Use of infusions of FIBRINOLYTIC AGENTS to destroy or dissolve thrombi in blood vessels or bypass grafts.
The restoration of blood supply to the myocardium. (From Dorland, 28th ed)
Drugs that bind to but do not activate beta-adrenergic receptors thereby blocking the actions of beta-adrenergic agonists. Adrenergic beta-antagonists are used for treatment of hypertension, cardiac arrhythmias, angina pectoris, glaucoma, migraine headaches, and anxiety.
Surgery performed on the heart.
The return of a sign, symptom, or disease after a remission.
A statistical technique that isolates and assesses the contributions of categorical independent variables to variation in the mean of a continuous dependent variable.
Studies to determine the advantages or disadvantages, practicability, or capability of accomplishing a projected plan, study, or project.
A method of studying a drug or procedure in which both the subjects and investigators are kept unaware of who is actually getting which specific treatment.
Hospital department responsible for the administration and provision of immediate medical or surgical care to the emergency patient.
Dilation of an occluded coronary artery (or arteries) by means of a balloon catheter to restore myocardial blood supply.
Removal of tissue with electrical current delivered via electrodes positioned at the distal end of a catheter. Energy sources are commonly direct current (DC-shock) or alternating current at radiofrequencies (usually 750 kHz). The technique is used most often to ablate the AV junction and/or accessory pathways in order to interrupt AV conduction and produce AV block in the treatment of various tachyarrhythmias.
A medical specialty concerned with the provision of continuing, comprehensive primary health care for the entire family.
Statistical models used in survival analysis that assert that the effect of the study factors on the hazard rate in the study population is multiplicative and does not change over time.
A nonparametric method of compiling LIFE TABLES or survival tables. It combines calculated probabilities of survival and estimates to allow for observations occurring beyond a measurement threshold, which are assumed to occur randomly. Time intervals are defined as ending each time an event occurs and are therefore unequal. (From Last, A Dictionary of Epidemiology, 1995)
A graphic means for assessing the ability of a screening test to discriminate between healthy and diseased persons; may also be used in other studies, e.g., distinguishing stimuli responses as to a faint stimuli or nonstimuli.
The proportion of survivors in a group, e.g., of patients, studied and followed over a period, or the proportion of persons in a specified group alive at the beginning of a time interval who survive to the end of the interval. It is often studied using life table methods.
A group of pathological conditions characterized by sudden, non-convulsive loss of neurological function due to BRAIN ISCHEMIA or INTRACRANIAL HEMORRHAGES. Stroke is classified by the type of tissue NECROSIS, such as the anatomic location, vasculature involved, etiology, age of the affected individual, and hemorrhagic vs. non-hemorrhagic nature. (From Adams et al., Principles of Neurology, 6th ed, pp777-810)
Small-scale tests of methods and procedures to be used on a larger scale if the pilot study demonstrates that these methods and procedures can work.
Organized periodic procedures performed on large groups of people for the purpose of detecting disease.
The number of males and females in a given population. The distribution may refer to how many men or women or what proportion of either in the group. The population is usually patients with a specific disease but the concept is not restricted to humans and is not restricted to medicine.
Physical activity which is usually regular and done with the intention of improving or maintaining PHYSICAL FITNESS or HEALTH. Contrast with PHYSICAL EXERTION which is concerned largely with the physiologic and metabolic response to energy expenditure.
Procedures for finding the mathematical function which best describes the relationship between a dependent variable and one or more independent variables. In linear regression (see LINEAR MODELS) the relationship is constrained to be a straight line and LEAST-SQUARES ANALYSIS is used to determine the best fit. In logistic regression (see LOGISTIC MODELS) the dependent variable is qualitative rather than continuously variable and LIKELIHOOD FUNCTIONS are used to find the best relationship. In multiple regression, the dependent variable is considered to depend on more than a single independent variable.
The thoracolumbar division of the autonomic nervous system. Sympathetic preganglionic fibers originate in neurons of the intermediolateral column of the spinal cord and project to the paravertebral and prevertebral ganglia, which in turn project to target organs. The sympathetic nervous system mediates the body's response to stressful situations, i.e., the fight or flight reactions. It often acts reciprocally to the parasympathetic system.
An infant during the first month after birth.
The presence of co-existing or additional diseases with reference to an initial diagnosis or with reference to the index condition that is the subject of study. Comorbidity may affect the ability of affected individuals to function and also their survival; it may be used as a prognostic indicator for length of hospital stay, cost factors, and outcome or survival.
The number of new cases of a given disease during a given period in a specified population. It also is used for the rate at which new events occur in a defined population. It is differentiated from PREVALENCE, which refers to all cases, new or old, in the population at a given time.
Abrupt changes in the membrane potential that sweep along the CELL MEMBRANE of excitable cells in response to excitation stimuli.
Naturally occurring or experimentally induced animal diseases with pathological processes sufficiently similar to those of human diseases. They are used as study models for human diseases.

Investigation of the theory and mechanism of the origin of the second heart sound. (1/14381)

To investigate further the origin of the second heart sound we studied human subjects, dogs, and a model in vitro of the cardiovascular system. Intra-arterial sound, pressure, and, where possible, flow and high speed cine (2,000 frames/sec) were utilized. The closure sound of the semilunar valves was of higher amplitude in be ventricles than in their respective arterial cavities. The direction of inscription of the main components of intra-arterial sound were opposite in direction to the components of intraventricular sound. Notches, representative of pressure increments, were noted on the ventricular pressure tracings and were coincident with the components of sound. The amplitude of the closure sound varied with diastolic pressure, but remained unchanged with augmentation of forward and retrograde aortic flow. Cines showed second sound to begin after complete valvular closure, and average leaflet closure rate was constant regardless of pressure. Hence, the semilunar valves, when closed, act as an elastic membrane and, when set into motion, generate compression and expansion of the blood, producing transient pressure changes indicative of sound. The magnitude of the initial stretch is related to the differential pressure between the arterial and ventricular chambers. Sound transients which follow the major components of the second sound appear to be caused by the continuing stretch and recoil of the leaflets. Clinically unexplained findings such as the reduced or absent second sound in calcific aortic stenosis and its paradoxical presence in congenital aortic stenosis may be explained by those observations.  (+info)

Hierarchy of ventricular pacemakers. (2/14381)

To characterize the pattern of pacemaker dominance in the ventricular specialized conduction system (VSCS), escape ventricular pacemakers were localized and quantified in vivo and in virto, in normal hearts and in hearts 24 hours after myocardial infarction. Excape pacemaker foci were localized in vivo during vagally induced atrial arrest by means of electrograms recorded from the His bundle and proximal bundle branches and standard electrocardiographic limb leads. The VSCS was isolated using a modified Elizari preparation or preparations of each bundle branch. Peacemakers were located by extra- and intracellular recordings. Escape pacemaker foci in vivo were always in the proximal conduction system, usually the left bundle branch. The rate was 43+/-11 (mean+/-SD) beats/min. After beta-adrenergic blockade, the mean rate fell to 31+/-10 beats/min, but there were no shifts in pacemaker location. In the infarcted hearts, pacemakers were located in the peripheral left bundle branch. The mean rate was 146+/-20 beats/min. In isolated normal preparations, the dominant pacemakers usually were in the His bundle, firing at a mean rate of 43+/-10 beats/min. The rates of pacemakers diminished with distal progression. In infarcted hearts, the pacemakers invariably were in the infarct zone. The mean firing rates were not influenced by beta-adrenergic blockade. The results indicate that the dominant pacemakers are normally in the very proximal VSCS, but after myocardial infarction pacemaker dominance is shifted into the infarct. Distribution of pacemaker dominance is independent of sympathetic influence.  (+info)

Irbesartan reduces QT dispersion in hypertensive individuals. (3/14381)

Angiotensin type 1 receptor antagonists have direct effects on the autonomic nervous system and myocardium. Because of this, we hypothesized that irbesartan would reduce QT dispersion to a greater degree than amlodipine, a highly selective vasodilator. To test this, we gathered electrocardiographic (ECG) data from a multinational, multicenter, randomized, double-blind parallel group study that compared the antihypertensive efficacy of irbesartan and amlodipine in elderly subjects with mild to moderate hypertension. Subjects were treated for 6 months with either drug. Hydrochlorothiazide and atenolol were added after 12 weeks if blood pressure (BP) remained uncontrolled. ECGs were obtained before randomization and at 6 months. A total of 188 subjects (118 with baseline ECGs) were randomized. We analyzed 104 subjects who had complete ECGs at baseline and after 6 months of treatment. Baseline characteristics between treatments were similar, apart from a slight imbalance in diastolic BP (irbesartan [n=53] versus amlodipine [n=51], 99.2 [SD 3. 6] versus 100.8 [3.8] mm Hg; P=0.03). There were no significant differences in BP normalization (diastolic BP <90 mm Hg) between treatments at 6 months (irbesartan versus amlodipine, 80% versus 88%; P=0.378). We found a significant reduction in QT indexes in the irbesartan group (QTc dispersion mean, -11.4 [34.5] milliseconds, P=0.02; QTc max, -12.8 [35.5] milliseconds, P=0.01), and QTc dispersion did not correlate with the change in BP. The reduction in QT indexes with amlodipine (QTc dispersion, -9.7 [35.4] milliseconds, P=0.06; QTc max, -8.6 [33.2] milliseconds, P=0.07) did not quite reach statistical significance, but there was a correlation between the change in QT indexes and changes in systolic BP. In conclusion, irbesartan improved QT dispersion, and this effect may be important in preventing sudden cardiac death in at-risk hypertensive subjects.  (+info)

QT dispersion in patients with chronic heart failure: beta blockers are associated with a reduction in QT dispersion. (4/14381)

OBJECTIVE: To compare QT dispersion in patients with impaired left ventricular systolic function and in matched control patients with normal left ventricular systolic function. DESIGN: A retrospective, case-control study with controls matched 4:1 for age, sex, previous myocardial infarction, and diuretic and beta blocker treatment. SETTING: A regional cardiology centre and a university teaching hospital. PATIENTS: 25 patients with impaired left ventricular systolic function and 100 patients with normal left ventricular systolic function. MAIN OUTCOME MEASURES: QT and QTc dispersion measured by three methods: the difference between maximum and minimum QT and QTc intervals, the standard deviation of QT and QTc intervals, and the "lead adjusted" QT and QTc dispersion. RESULTS: All measures of QT/QTc dispersion were closely interrelated (r values 0.86 to 0.99; all p < 0.001). All measures of QT and QTc dispersion were significantly increased in the patients with impaired left ventricular systolic function v controls (p < 0.001): 71.9 (6.5) (mean (SEM)) v 46.9 (1.7) ms for QT dispersion, and 83.6 (7.6) v 54.3 (2.1) ms(-1-2) for QTc dispersion. All six dispersion parameters were reduced in patients taking beta blockers (p < 0.05), regardless of whether left ventricular function was normal or impaired-by 9.4 (4.6) ms for QT dispersion (p < 0.05) and by 13.8 (6. 5) ms(-1-2) for QTc dispersion (p = 0.01). CONCLUSIONS: QT and QTc dispersion are increased in patients with systolic heart failure in comparison with matched controls, regardless of the method of measurement and independently of possible confounding factors. beta Blockers are associated with a reduction in both QT and QTc dispersion, raising the possibility that a reduction in dispersion of ventricular repolarisation may be an important antiarrhythmic mechanism of beta blockade.  (+info)

Tachyarrhythmias triggered by swallowing and belching. (5/14381)

Three cases with supraventricular tachyarrhythmias related to oesophageal transit are reported. A 61 year old man had episodes of atrial tachycardia on each swallow of food but not liquid; this has been reported only rarely. A 55 year old man had atrial fibrillation initiated by drinking ice cold beverages; this has not been described previously although atrial tachycardia triggered by drinking ice cold beverages has been described once. A 68 year old man had supraventricular tachycardia initiated by belching; this has not been described previously. These cases illustrate the diversity of atrial tachyarrhythmias that can be precipitated by oesophageal stimulation and suggest that what is regarded as a very rare phenomenon may be found more commonly when sought.  (+info)

Tachycardia induced tachycardia: case report of right ventricular outflow tract tachycardia and AV nodal reentrant tachycardia. (6/14381)

Tachycardia induced tachycardia, or so called double tachycardia, is rare. A 34 year old woman is described who had a history of syncope, frequent extrasystoles, and episodes of non-sustained ventricular tachycardia, perceived as palpitation, without syncope. At electrophysiological study, during infusion of isoprenaline, an episode of non-sustained ventricular tachycardia arising from the right ventricular outflow tract initiated sustained atrioventricular nodal reentrant tachycardia, thought to be the cause of the patient's syncope. Ablation of the right ventricular outflow tract focus abolished the ventricular ectopy; the slow AV nodal pathway was also ablated. The patient no longer has either syncope or palpitation.  (+info)

ECG diagnosis of native heart ventricular tachycardia in a heterotopic heart transplant recipient. (7/14381)

A case is reported of haemodynamic collapse in a 51 year old male heterotopic heart transplant recipient caused by native heart ventricular tachycardia. An accurate diagnosis was made by selective right and left sided electrocardiography. Synchronised electrical cardioversion of the native heart (200 J) resulted in restoration of sinus rhythm with prompt relief of symptoms and amelioration of the clinical situation.  (+info)

Prognostic value of myocardial perfusion imaging in patients with high exercise tolerance. (8/14381)

BACKGROUND: Although high exercise tolerance is associated with an excellent prognosis, the significance of abnormal myocardial perfusion imaging (MPI) in patients with high exercise tolerance has not been established. This study retrospectively compares the utility of MPI and exercise ECG (EECG) in these patients. METHODS AND RESULTS: Of 388 consecutive patients who underwent exercise MPI and reached at least Bruce stage IV, 157 (40.5%) had abnormal results and 231 (59.5%) had normal results. Follow-up was performed at 18+/-2.7 months. Adverse events, including revascularization, myocardial infarction, and cardiac death, occurred in 40 patients. Nineteen patients had revascularization related to the MPI results or the patient's condition at the time of MPI and were not included in further analysis. Seventeen patients (12.2%) with abnormal MPI and 4 (1.7%) with normal MPI had adverse cardiac events (P<0.001). Cox proportional-hazards regression analysis showed that MPI was an excellent predictor of cardiac events (global chi2=13.2; P<0.001; relative risk=8; 95% CI=3 to 23) but EECG had no predictive power (global chi2=0.05; P=0.8; relative risk=1; 95% CI=0.4 to 3.0). The addition of Duke's treadmill score risk categories did not improve the predictive power of EECG (global chi2=0.17). The predictive power of the combination of EECG (including Duke score categories) and MPI was no better than that of MPI alone (global chi2=13.5). CONCLUSIONS: Unlike EECG, MPI is an excellent prognostic indicator for adverse cardiac events in patients with known or suspected CAD and high exercise tolerance.  (+info)

This ECG was obtained from a man in his 70s. We have no other clinical information. It is interesting for several reasons. Giant T wave inversions The most obvious abnormalities we see on first inspection are the deeply inverted T waves in Leads V3 through V6. The T wave in V3 is biphasic. There are also T wave inversions in all of the limb leads except aVR. The precordial T wave inversions are called giant T wave inversions because they are 10 mm or more in depth. There are many causes of giant T wave inversions, including, but not limited to: myocardial ischemia, coronary artery disease and reperfusion, pulmonary edema, massive pulmonary embolism, subarachnoid hemorrhage, apical hypertrophy, post-tachycardia syndrome, and post-pacing syndrome. What else? There are no Q waves or ST elevations. The ST segments are not entirely normal in shape, being flattened in most lead. The frontal plane axis is left. Even though the ECG almost meets criteria for left ventricular hypertrophy, by exclusion ...
Purpose T wave inversion (TWI) is the electrical hallmark of cardiac conditions such as hypertrophic cardiomyopathy (HCM) or arrhythmogenic right ventricular cardiomyopathy (ARVC), which may be the substrate for sudden cardiac death in the young athlete. Such repolarization anomalies can feature on the ECG of an apparently healthy athlete and pose major diagnostic dilemmas in sports cardiology, as regular, prolonged high intensity, physical activity is associated with such repolarization changes. Athletes themselves are reluctant to detrain during the season, which makes interpreting any reversible effects of exercise on the ECG more difficult. This study aimed to investigate the effect of detraining on TWI in athletes. ...
Introduction: While right precordial T wave inversions (RPTWI) are associated with specific cardiomyopathic conditions including ARVC, they are common among trained athletes without underlying heart disease. RPTWI in athletes, particularly those that are not of Afro-Caribbean descent, remain poorly understood and represent a source of considerable clinical controversy. We therefore sought to examine the prevalence, training physiology specificity, and ECG correlates of RPTWI among asymptomatic Caucasian athletes.. Methods: Competitive collegiate Caucasian male endurance athletes (rowers, n=168) and strength-trained athletes (football players, n=162) underwent standard 12-lead ECG at the time of pre-participation screening. ECGs were analyzed for standard quantitative parameters and for the presence of RPTWI defined as TWI ≥2mm in ≥2 leads from V1 to V3.. Results: RPTWI were significantly more prevalent in endurance athletes (25%) than in strength-trained athletes (4%, p,0.001; Figure). ...
Introduction: While it has been proposed that T wave inversions (TWI) in the anterior precordial leads can be a normal finding in the ECGs of Afro-Caribbean athletes, it is uncertain whether this holds true for African-Americans. Hypothesis: TWI in the anterior precordial leads can be a non-specific marker of cardiac disease, and as a result, assuming a benign nature for TWI in the anterior leads in African-American athletes may not be appropriate. Methods: To begin to investigate this notion, we evaluated the incidence of cardiovascular death (CVD) in apparently healthy African- Americans with anterior TWI over an 11 year period. We analyzed the ECGs and CV deaths in 5334 ambulatory African Americans (average age 50 years, 8% female, average follow up of 8 years) seen at the Palo Alto VA Health Care system from 1986 until 1997. T waves were coded as inverted in V2, V3, V4 and V5 if TWI were noted to be more than 1 mm below the PR segment. The leads coded as inverted were summed to create a ...
OBJECTIVE: To investigate the value of a giant negative T wave (, or = 1.0 mV) in precordial leads of 12-lead electrocardiograms in the acute phase of Q wave myocardial infarction as a predictor of myocardial salvage. METHODS: Coronary angiographic and electrocardiographic findings, left ventricular ejection fraction in the chronic stage, and levels of cardiac enzymes were compared in patients with myocardial infarction with (group GNT, n = 31) and without (group N, n = 20) a giant negative T wave. GNT patients were divided into two subgroups according to the presence (GNT:R[+], n = 10) or absence (GNT: R[-], n = 21) of R wave recovery with an amplitude , or = 0.1 mV in at least one lead that had shown Q waves. RESULTS: The maximum level of creatine kinase and the total creatine kinase were lower in group GNT compared with group N (P , 0.05). The left ventricular ejection fraction was higher in group GNT than in group N (P , 0.05). The maximum creatine kinase and total creatine kinase were lower ...
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You systematically review the EKG. The heart rate is normal. There is a p wave before every QRS complex, but the p-wave axis is abnormal (negative in lead II). There is now an extreme rightward axis as the QRS is negative in leads I, II and avF. As you analyze the ST segments for evidence of ischemia, there appears to be T wave inversions in the inferior distribution of II, III, and avF. You start considering the differential of ectopic atrial rhythm, right axis deviation, and T wave inversions including PE or ischemia but stop yourself. You think did someone switch the leads? You take a brief look at aVR and see that the P, QRS and T waves are positive, making you even more suspicious ...
Alteration in ventricular repolarization occurs during changes in ventricular activation as may occur with ventricular pacing, bundle branch block and various arrhythmias. This may result in T wave abnormalities which persist following cessation of pacing, resolution of bundle branch block or resolution of arrhythmia. Such changes may be confused with ischemia. The phenomenon, known as cardiac memory, is reviewed here in the American Journal of Emergency Medicine. ...
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IF it would not be for the ST elevation in lead V6 - the principal finding on ECG #1 would be the ST-T wave depression in anterior (as well as right-sided) leads V1,V2,V3. Anterior ST depression and/or T wave inversion is an ECG finding that should always prompt consideration of RV strain, as occurs in acute PE (For more on ECG recognition of Acute PE - Please see My Comment at the bottom of Dr. Smiths April 5, 2019 Blog post). Other findings in ECG #1 that are potentially consistent with acute RV strain are the presence of numerous S waves (in leads I, II, III; and across all chest leads) + what could be construed as an incomplete RBBB pattern in lead V1 (with narrow terminal S waves in leads I and V6). But AGAINST acute PE as the diagnosis are: i) the History, which sounds cardiac, without hint of acute dyspnea; and, ii) the unusual ECG finding of ST-T wave depression being by far most remarkable in lead V1, and no more than modest in leads V2 and V3. Typically, with acute RV strain - ...
A method of gathering electrocardiographic data is disclosed wherein the EKG data is collected by sequentially monitoring the difference in electrical potential measured at each of at least three patient leads for a time period greater than 15 seconds, preferably for equal time periods of 20 seconds each. Although more than three leads and longer monitoring time periods may be used, a 60-second three-lead EKG data sample in accordance with the present invention is clinically sufficient to monitor cardiac function and to diagnose most cardiac dysfunctions. A system for collecting such electrocardiographic data is also disclosed. The system allows the collected data to be stored for later translation and interpretation. The stored data may also be communicated over phone lines for translation and interpretation at a remote site by trained individuals.
This webinar is designed for those staff who have experience in basic Lead II ECG interpretation and wish to learn how to analyze and interpret 12 lead ECGs. The workshop will focus on the need to know subjects of 12 Lead Electrocardiography and will include basic electrophysiology, a framework for interpretation, a review of the normal 12 Lead ECG, abnormalities, and recognition of ECG changes seen in myocardial ischemia and infarction ...
by ltwardzik829, Sep. 2005. EKG Test Prep EKG Technician Exam Practice Test Questions. It is likely that in many clinical settings youll be asked to regularly review ECGs. Write. Help us carry on by supporting the project. Ekg National Exam Study Guide , datacenterdynamics.com Prepare for your Certified EKG Technician (CET) exam with the comprehensive 106-page printed study guide and set of three different online practice tests. EKG Technician Exam , AMCA EKG Technician Certification Exam Review , Udemy Free Phlebotomy Practice Tests 2020 [100+ Questions] Certified EKG Technician (CET) Exam Application - Page 1/27. EKG practice test. Jump to Page . EKG Certification Exam study guide!! Doctoral Degree. You are on page 1 of 19. Match. This book is a bank of 350+ questions to prepare for the EKG Technician national certification exams. K. Lee, MD. Search. Get Free Ekg Tech Practice Test now and use Ekg Tech Practice Test immediately to get % off or $ off or free shipping. Epsilon a ...
In this chapter, we investigate the most recent automatic detecting algorithms on abnormal electrocardiogram (ECG) in a variety of cardiac arrhythmias. We present typical examples of a medical case study and technical applications related to diagnosing ECG, which include (i) a recently patented data classifier on the basis of deep learning model, (ii) a deep neural network scheme to diagnose variable types of arrhythmia through wearable ECG monitoring devices, and (iii) implementation of the health cloud platform, which consists of automatic detection, data mining, and classifying via the Android terminal module. Our work establishes a cross-area study, which relates artificial intelligence (AI), deep learning, cloud computing on huge amount of data to minishape ECG monitoring devices, and portable interaction platforms. Experimental results display the technical advantages such as saving cost, better reliability, and higher accuracy of deep learning-based models in contrast to conventional schemes on
ECG in hypertrophic cardiomyopathy with atrial fibrillation: Atrial activity is seen as irregular fibrillary waves suggesting atrial fibrillation. The QRS complexes have a large amplitude in chest leads overlapping between the leads. Tall R waves in lateral leads and deep S waves in anterior leads along with gross ST segment depression with T wave inversion in lateral leads are suggestive of severe left ventricular hypertrophy. The QRS width is also increased to about 120 msec mimicking left bundle branch block. The ECG is from a case of advanced hypertrophic cardiomyopathy with atrial fibrillation. Development of atrial fibrillation leads to cardiac decompensation in hypertrophic cardiomyopathy due to loss of atrial kick. In a hypertrophied ventricle the booster effect of atrial contraction is very important for diastolic filling. Even though the contribution of atrial contraction to ventricular filling in a normal person is about 15 - 20 %, it may be over 30% in an individual with diastolic ...
The discrepancy between the relatively better early outcomes and the similar or even worse long-term prognoses in patients with non-Q wave versus Q wave infarction has led to a more aggressive approach in the management of this group of patients ([3, 5, 10]). Our findings conflict somewhat with reports from the prethrombolytic era ([3-5, 9, 17-19]), in which 1-year mortality was similar or even higher in the non-Q wave infarction populations. The difference could be related to the heterogeneity of the populations, the definitions used and the use of thrombolysis ([3-9, 11]).. Another possible explanation for this disagreement is the initial ST segment deviation. In most studies of Q wave or non-Q wave infarction, the initial ST segment shift was seldom considered a prognostic marker. The observation that patients with initial ST segment depression have a worse prognosis than those with ST segment elevation and either Q wave or non-Q wave infarction ([20-23]), with the additional evidence that ...
The correlation between persistent negative T wave on basal electrocardiogram and coronary anatomy or global and regional left ventricular function was investigated in 34 patients with unstable angina defined as new onset (| 2 months), crescendo or rest angina. The patients with history of previous myocardial infarction, pathological Q waves on electrocardiogram or documented elevation of CPK were excluded. Eighteen patients (group A) showed T wave inversion (| 1 mV) in at least two leads on the basal electrocardiogram, persisting for at least 48 hours before coronary arteriography. In 16 patients (group B) the basal electrocardiogram was normal. Left ventricular volumes and ejection fraction were calculated and the regional systolic wall motion was analyzed using the area method in the 30 degrees right anterior oblique view. Hypokinesis was defined as more than 2 standard deviation below the mean value calculated in 24 normal subjects. No difference was present for age (A: 61 +/- 9 vs B: 57 +/- 9 yrs)
Is there a ECG marker for recent syncope ? Yes . This was classically described many decades ago. Following a Stokes -Adam attack when the patient recovers from the loss of consciousness a peculiar ECG pattern was observed. A typical ECG from our CCU The mechanism is not clear.It can be due to 1. Repolarisation…
GREAT case! My initial impression looking at the initial ECG was that the leads (especially leads III and aVF) just looked funny - almost as if there was some type of lead misplacement. That is, the amount of T wave inversion (especially in lead III) looked extreme - albeit both QRS and ST-T wave appearance in lead III was indeed the precise mirror-image of the QRST & ST-T wave in lead aVL …But there is no lead misplacement - as the P wave in lead II is upright, and there is global negativity in lead aVR as there is expected to be. Looking next at the chest leads - not only R wave progression, but also R wave amplitude looks uncharacteristically reduced - which if not due to large body habitus (I dont think this patients body habitus was described … ?) should raise the question if loss of R wave on this initial ECG is the result of an acute ongoing event. Regardless of questions raised by these findings - there is little doubt (as recognized by the treating ED physician) that T wave ...
Background:. The importance of ischemic ECG changes including St segment elevation, ST segment depression or T wave inversion that indicate myocardial ischemia are well established and require appropriate investigation and treatment.. However, there is an abundance of clinical situation, with apparently ischemic ECG change not indicate traditionally coronary artery related ischemia and therefore require prompt recognition and treatment of underlying condition, that may be serious and life-threatened. For example of such conditions are pericarditis, myocarditis, aortic dissection, electrolyte abnormalities, intracranial hemorrhage and hypothermia.. Together with them, an ECG ST segment changes may appeared in abdominal serious illness such as pancreatitis and cholecystitis(17,18,19,20,23). Whereas in pancreatitis various vasoactive and toxic for myocardium substances released, the cause of ST segment changes in cholecystitis are discussed and includes tachycardia , vagal reflexes, changed in ...
Background:. The importance of ischemic ECG changes including St segment elevation, ST segment depression or T wave inversion that indicate myocardial ischemia are well established and require appropriate investigation and treatment.. However, there is an abundance of clinical situation, with apparently ischemic ECG change not indicate traditionally coronary artery related ischemia and therefore require prompt recognition and treatment of underlying condition, that may be serious and life-threatened. For example of such conditions are pericarditis, myocarditis, aortic dissection, electrolyte abnormalities, intracranial hemorrhage and hypothermia.. Together with them, an ECG ST segment changes may appeared in abdominal serious illness such as pancreatitis and cholecystitis(17,18,19,20,23). Whereas in pancreatitis various vasoactive and toxic for myocardium substances released, the cause of ST segment changes in cholecystitis are discussed and includes tachycardia , vagal reflexes, changed in ...
Early repolarization pattern (ERP) on the 12-lead electrocardiogram (ECG) is associated with sudden cardiac death (SCD) and increased mortality in certain populations. The mechanism of this association has been hypothesized to relate to regional heterogeneity of epicardial and endocardial repolarization. However, whether differences in subclinical left ventricular wall thickness may affect this association is unknown (1). Individuals with ERP are more likely to meet Sokolow-Lyon ECG criteria for left ventricular hypertrophy (LVH-SL) but are paradoxically less likely to meet Cornell voltage criteria (LVH-C) (2,3). Studies to date are limited by use of ECG measurements to define LVH and are thus not sufficient to determine whether there is a relationship between left ventricular mass (LVM) and ERP. We hypothesized that participants with ERP have a higher LVM and a greater prevalence of cardiac magnetic resonance imaging-determined LVH (LVH-CMR) compared with those without ERP.. The ...
Have recently had ECG done following chest pain.Tracing showed T wave inversion and ST wave inversion in lead 3. Have then had normal Echo, stress ECG and 24 hour Holter monitor. What could be the reas...
Mesocardia & Normal Electrocardiogram & Vascular Disease Symptom Checker: Possible causes include Corrected Transposition of the Great Vessels. Check the full list of possible causes and conditions now! Talk to our Chatbot to narrow down your search.
Intraventricular conduction delays such as LBBB and the associated ST segment-T wave abnormalities can mimic both acute and chronic ischaemic changes. Much has been written about the evaluation of the ST segment elevation in the presence of LBBB1,8; considering chest pain patients in the ED, LBBB is responsible for 15% of STE syndromes and is the second most frequently encountered electrocardiographic pattern responsible for non-ischaemic STE.17,18 LBBB, however, can also cause significant ST segment depression, and it is imperative that these electrocardiographic changes be distinguished from those that occur in the presence of ACS.3 The rule of appropriate discordance states that in LBBB, ST segment-T wave configurations are directed opposite from the major, terminal portion of the QRS complex. As such, leads with either QS or rS complexes should have significantly elevated ST segments mimicking an AMI while leads with a large monophasic R wave demonstrate ST segment depression. T waves in ...
article{3004d42f-c464-4188-bf60-ba0491925d72, author = {Bennhagen, Rolf and Sörnmo, Leif and Pahlm, Olle and Pesonen, Erkki}, issn = {1399-3046}, language = {eng}, number = {6}, pages = {773--779}, publisher = {Wiley-Blackwell}, series = {Pediatric Transplantation}, title = {Serial signal-averaged electrocardiography in children after cardiac transplantation.}, url = {http://dx.doi.org/10.1111/j.1399-3046.2005.00384.x}, volume = {9}, year = {2005 ...
This comprehensive 50 hour EKG Technician Certification Program prepares students to function as an EKG Technician. This course will include important practice and background information on anatomy of the heart and physiology, medical disease processes, medical terminology, medical ethics, legal aspects of patient contact, the Holter monitor, electrocardiography and echocardiography.. Additionally, students will practice with equipment and perform hands-on labs including introduction to the function and proper use of the EKG machine, the normal anatomy of the chest wall for proper lead placement, 12-lead placement and other clinical practices. EKG Technicians also analyze printed readings of EKG tests, measuring various cardiac intervals and complexes and determining normal vs. abnormal EKG.. EKG Technician Course Outline. ...
The QED 2000 is a complete 12 Lead Electrocardiograph (ECG) solution with ECG interpretation software. Designed to assist health care professionals in delivering the utmost quality care to patients, the ECG reading can be easily and accurately acquired, analyzed, stored and printed in one touch. With features designed to enhance portability, streamline workflow and assist in a physicians diagnosis, the QED 2000 12 Lead ECG is suitable for many environments.. The built-in ECG interpretation software assists in healthcare professionals diagnosis and screening. Our patented ECG interpretation software is a unique algorithm which analyzes the frequency components of a 10 second 12 lead resting ECG reading. Clinically tested and with a high sensitivity/specificity rate, the ECG interpretation offers a reliable and accurate analysis to support a physicians diagnosis. Segment and waveform measurement, rhythm interpretation, and myocardial ischemia sensitivity probability is generated by the QED 2000 ...
A 42-year-old man with acute renal failure is confused. His serum potassium is 8.1 mEq/L . The most likely abnormal ECG finding is: a) T wave inversion b) PR interval of 300ms c) QT interval of 0.4s d) U wave e) Tall tented T waves The correct answer is E The earliest ECG evidence of [...]. ...
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Here is a good organized approach to master ECG interpretation. Learning the art of ECG interpretation requires intellect, commitment, effort and - perhaps most importantly - an organized approach.
Signal-averaged electrocardiography (SAECG) is a special electrocardiographic technique, in which multiple electric signals from the heart are averaged to remove interference and reveal small variations in the QRS complex, usually the so-called late potentials. These may represent a predisposition towards potentially dangerous ventricular tachyarrhythmias. A resting electrocardiogram (ECG) is recorded in the supine position using an ECG machine equipped with SAECG software; this can be done by a physician, nurse, or medical technician. Unlike standard basal ECG recording, which requires only a few seconds, SAECG recording requires a few minutes (usually about 7-10 minutes), as the machine must record multiple subsequent QRS potentials to remove interference due to skeletal muscle and to obtain a statistically significant average trace. For this reason, it is important for the patient to lie as still as possible during the recording. SAECG recording yields a single, averaged QRS potential, ...
Hi Mostafa. Any other lead area(s) of the heart may show reciprocal ST depression that opposes ST elevation in another area ... so to my knowledge there is no limitation as to which leads may show this. That said, there are certain common patterns. For example - with inferior wall acute STEMI - you will virtually always see a mirror image ( = reciprocal) shape of ST depression in lead aVL compared to the pattern of ST elevation that you see in lead III. And, in high lateral infarction when you have ST elevation in aVL - you will often see a mirror-image of ST depression in the inferior leads (esp. in lead III). Proximal LAD occlusions often show reciprocal ST depression in the inferior leads - but when the LAD occlusion is more distal, you may not see any reciprocal inferior ST depression at all .... ST depression in V1,V2,V3 in association with acute inferior STEMI may reflect reciprocal ST depression, but could also be due to acute posterior infarction or to anterior ischemia. So - ...
TY - JOUR. T1 - Seizure-related cardiac repolarization abnormalities are associated with ictal hypoxemia. AU - Seyal, Masud. AU - Pascual, Franchette. AU - Lee, Chia Yuan Michael. AU - Li, Chin-Shang. AU - Bateman, Lisa M.. PY - 2011/11. Y1 - 2011/11. N2 - Purpose: Cardiac arrhythmias and respiratory disturbances have been proposed as likely causes for sudden unexpected death in epilepsy. Oxygen desaturation occurs in one-third of patients with localization-related epilepsy (LRE) undergoing inpatient video-electroencephalography (EEG) telemetry (VET) as part of their presurgical workup. Ictal-related oxygen desaturation is accompanied by hypercapnia. Both abnormal lengthening and shortening of the corrected QT interval (QTc) on electrocardiography (ECG) have been reported with seizures. QTc abnormalities are associated with increased risk of sudden cardiac death. We hypothesized that there may be an association between ictal hypoxemia and cardiac repolarization abnormalities. Methods: VET data ...
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QRS width and morphology is in a gray zone. Precise answer really requires review after conversion to sinus rhythm. That said, the QRS looks supraventricular. While one can debate if QRS width is 0.11 vs 0.12 ... the width of the monophasic R wave in lateral leads is not overly wide; the initial r in V1,2,3 is larger than is usual in lbbb; and the QRS just looks supraventricular. Remember that BOTH LAHB and LVH may slightly widen the QRS - and that is my strong hunch of what we have here - :). Delete ...
Even though the electrocardiogram (ECG) is the standard diagnostic test for the evaluation of symptoms of acute myocardial ischemia or infarction, the prognostic value of various ECG features of cardiac ischemia is ill-defined. Savonitto and colleagues conducted a retrospective study of the ECG findings among patients in the Global Use of Strategies to Open Occluded Arteries in Acute Coronary Syndromes (GUSTO-IIb) trial.. A total of 12,142 patients were enrolled in the GUSTO-IIb trial. To be enrolled in the study, patients had to have ECG signs of myocardial ischemia, including transient or persistent ST-segment elevation or depression of more than 0.05 mV or persistent and definite T-wave inversion of more than 0.1 mV. All patients had to have reported symptoms of cardiac ischemia at rest within 12 hours of hospital admission. Since the GUSTO-IIb trial was a comparison of heparin and desirudin, patients were excluded if they had active bleeding, a history of stroke, an elevated serum creatinine ...
TY - JOUR. T1 - Fragmented QRS on twelve-lead electrocardiogram predicts arrhythmic events in patients with ischemic and nonischemic cardiomyopathy. AU - Das, Mithilesh. AU - Maskoun, Waddah. AU - Shen, Changyu. AU - Michael, Mark A.. AU - Suradi, Hussam. AU - Desai, Mona. AU - Subbarao, Roopa. AU - Bhakta, Deepak. PY - 2010/1. Y1 - 2010/1. N2 - Background: Myocardial scar is a substrate for reentrant ventricular arrhythmias and is associated with poor prognosis. Fragmented QRS (fQRS) on 12-lead ECG represents myocardial conduction delays due to myocardial scar in patients with coronary artery disease (CAD). Objective: The purpose of this study was to determine whether fQRS is associated with increased ventricular arrhythmic event and mortality in patients with CAD and nonischemic dilated cardiomyopathy (DCM). Methods: Arrhythmic events and mortality were studied in 361 patients (91% male, age 63.3 ± 11.4 years, mean follow-up 16.6 ± 10.2 months) with CAD and DCM who received an implantable ...
Looking for online definition of electrocardiographically in the Medical Dictionary? electrocardiographically explanation free. What is electrocardiographically? Meaning of electrocardiographically medical term. What does electrocardiographically mean?
Commercial. The crucial information on Electrocardiograph market size, geographical presence, the market share of top players is presented in this report. The report begins with the analysis of Electrocardiograph overview, objectives, market scope, and market size estimation. The past, present and forecast Electrocardiograph market scenario is presented with the market concentration and market saturation analysis. The market study outcomes are based on extensive primary and secondary research with the key opinion leaders of Electrocardiograph industry. The forecast Electrocardiograph growth trajectory is presented for the year 2020 to 2027 which will shape the development plans.. The regional analysis of Global Electrocardiograph Market is considered for the key regions such as Asia Pacific, North America, Europe, Latin America and Rest of the World. The Electrocardiograph market drivers, emerging segments, industry rules and regulations along with the development plans and policies are ...
Arrhythmogenic right ventricular cardiomyopathy/dysplasia clinical presentation and diagnostic evaluation: results from the North American Multidisciplinary Study.
TY - JOUR. T1 - Site-specific twelve-lead ECG features to identify an epicardial origin for left ventricular tachycardia in the absence of myocardial infarction. AU - Bazan, Victor. AU - Gerstenfeld, Edward P.. AU - Garcia, Fermin C.. AU - Bala, Rupa. AU - Rivas, Nuria. AU - Dixit, Sanjay. AU - Zado, Erica. AU - Callans, David J.. AU - Marchlinski, Francis E.. PY - 2007/11. Y1 - 2007/11. N2 - Background: Identification of an epicardial origin for left ventricular tachycardia (LV-VT) based on electrocardiogram (ECG) criteria facilitates the approach to catheter ablation. Reported criteria, although helpful, may not apply uniformly to all LV regions. Objective: We hypothesized that unique region-specific ECG patterns identify epicardial LV-VTs in patients without myocardial infarction. Methods: The QRS morphologies during pace mapping from 402 epicardial and 234 comparable endocardial sites and 19 epicardial VTs were analyzed in 15 patients with respect to morphology and duration of all and ...
Question - ECG:MI(lateral), left ventr. hypertrophy, left anterior hemiblock, left axis deviation, T-wave near baseline (lateral,inferior), -small positive T wave (anterior), -S waves up to V6, abnormal ECG. Ask a Doctor about diagnosis, treatment and medication for Hypertension, Ask a Cardiologist
Left bundle branch blocks. In left bundle branch block (LBBB) the left ventricle is not directly activated by impulses travelling through the left bundle branch. The right ventricle, however, is still activated as normal by the right bundle branch.. The left ventricle is activated by impulses travelling through the myocardium across the septum. As this occurs more slowly than conduction through the bundle of His the QRS complex becomes widened.. Normally the septum is activated from left to right, which produces small Q waves in the lateral leads. In the presence of LBBB, however, this septal activation is reversed, which eliminates these normal septal Q waves.. The right to left depolarization of the myocardium produces deep S waves in the right praecordial leads (V1-V3) and tall R waves in the lateral leads (I, V5 and V6). It also usually causes left axis deviation. As the ventricles are activated sequentially from right to left, rather than simultaneously, the R wave in the lateral leads is ...
Agree with lead placement errors with aVR being upright. However, P wave inversion can be seen in dextrocardia as can an upright in aVR. In addition p wave inversion can be seen in junctional rythm with retrograde conduction, T wave inversion in inferior leads could indicate ischemia. So, 1st check leads, if correct would perform an ECHO, which could detect both dextrocardia and wall motion abnormalities of inferior ischemia, if normal consider EP study. Most likely leads, then consider dextrocardia, then inferior ischemia then junctional with retrograde P ...
Can left bundle branch block cause chest pain? Mammen Ninan, Jonathan W Swan Exercise-induced left bundle branch block usually indicates underlying coronary artery disease or myocardial disease. Association of left bundle branch block (LBBB) with chest pain in the absence of coronary artery disease is rare. We describe the case history of a patient with chest pain associated with left bundle branch block with normal coronary arteries and review the literature on left bundle branch block associated with chest pain.. ...
Sakamoto T, Tei C, Murayama M, Ichiyasu H, Hada Y. Giant T wave inversion as a manifestation of asymmetrical apical hypertrophy (AAH) of the left ventricle: echocardiographic and ultrasono-cardiotomographic study. Jpn Heart J. 1976;17(5):611-29. PMID: 136532. DOI: https://doi.org/10.1536/ihj.17.611 Yamaguchi H, Ishimura T, Nishiyama S, Nagasaki F, Nakanishi S, Takatsu F, et al. Hypertrophic nonobstructive cardiomyopathy with giant negative T waves (apical hypertrophy): Ventriculographic and echocardiographic features in 30 patients. Am J Cardiol. 1979;44(3):401- 12. PMID: 573056. DOI: https://doi.org/10.1016/0002-9149(79)90388-6 Eriksson MJ, Sonnenberg B, Woo A, Rakowski P, Parker TG, Wigle ED, et al. Long-term outcome in patients with apical hypertropic cardiomyopathy. J Am Cardiol. 2002;39(4):638- 45. PMID: 11849863. DOI: https://doi.org/10.1016/s0735-1097(01)01778-8 Kitaoka H, Doi Y, Casey SA, Hitomi M, Furuno T, Maron BJ. Comparision of prevalence of apical hypertrophic cardiomyopathy in ...
The disease was first described by Giovanni Maria Lancisi in 1736, who in his book De Motu Cordis et Aneurysmatibus reported a family with disease recurrence in four generations: the affected members presented with palpitations, heart failure, dilation and aneurysms of the RV and sudden death [9].. Dalla Volta et al. in 1961 reported a patient with auricularization of the RV pressure curve, emphasizing the peculiar hemodynamic picture of this non-ischemic heart muscle disease with RV behaving like an atrium [10]. However, we had to wait until the 80s to find the first clinical and pathologic series of patients with ARVC/D reported by Drs Marcus, Nava and Thiene [1-3].. Marcus et al. in 1982 reported the disease in adults, first emphasizing the origin of arrhythmias from the RV and the histopathological substrate consisting of fibro-fatty replacement of the RV free wall, accounting for epsilon wave and ventricular arrhythmias of RV origin with left bundle branch block (LBBB) morphology ...
TY - JOUR. T1 - Clinical and genetic heterogeneity of right bundle branch block and ST-segment elevation syndrome. T2 - A prospective evaluation of 52 families. AU - Priori, Silvia G.. AU - Napolitano, Carlo. AU - Gasparini, Maurizio. AU - Pappone, Carlo. AU - Della Bella, Paolo. AU - Brignole, Michele. AU - Giordano, Umberto. AU - Giovannini, Tiziana. AU - Menozzi, Carlo. AU - Bloise, Raffaella. AU - Crotti, Lia. AU - Terreni, Liana. AU - Schwartz, Peter J.. PY - 2000/11/14. Y1 - 2000/11/14. N2 - Background - The ECG pattern of right bundle branch block and ST-segment elevation in leads V1 to V3 (Brugada syndrome) is associated with high risk of sudden death in patients with a normal heart. Current management and prognosis are based on a single study suggesting a high mortality risk within 3 years for symptomatic and asymptomatic patients alike. As a consequence, aggressive management (implantable cardioverter defibrillator) is recommended for both groups. Methods and Results - Sixty patients ...
TY - JOUR. T1 - Abnormal electrocardiographic findings in athletes. T2 - Recognising changes suggestive of cardiomyopathy. AU - Drezner, Jonathan A.. AU - Ashley, Euan. AU - Baggish, Aaron L.. AU - Börjesson, Mats. AU - Corrado, Domenico. AU - Owens, David S.. AU - Patel, Akash. AU - Pelliccia, Antonio. AU - Vetter, Victoria L.. AU - Ackerman, Michael J.. AU - Anderson, Jeff. AU - Asplund, Chad A.. AU - Cannon, Bryan C.. AU - DiFiori, John. AU - Fischbach, Peter. AU - Froelicher, Victor. AU - Harmon, Kimberly G.. AU - Heidbuchel, Hein. AU - Marek, Joseph. AU - Paul, Stephen. AU - Prutkin, Jordan M.. AU - Salerno, Jack C.. AU - Schmied, Christian M.. AU - Sharma, Sanjay. AU - Stein, Ricardo. AU - Wilson, Mathew. N1 - Copyright: Copyright 2013 Elsevier B.V., All rights reserved.. PY - 2013/2. Y1 - 2013/2. N2 - Cardiomyopathies are a heterogeneous group of heart muscle diseases and collectively are the leading cause of sudden cardiac death (SCD) in young athletes. The 12-lead ECG is utilised as ...
Electrocardiograph ( EKG ) Technicians work closely with cardiovascular technologists. Technicians who specialize in electrocardiogram (EKG) testing are known as cardiographic or electrocardiograph (EKG) technicians. Electrocardiograph ( EKG ) Technicians conduct procedures and manage equipment that tests, monitors and documents electrical activity of a patients cardiovascular system. These test are used to assist cardiologists and physicians in identifying and treating cardiac and blood vessel abnormalities. These procedures are non-invasive and usually conducted as part of a routine examination before surgical procedures. These types of test include standard electrocardiogram exams, 12-lead placement, cardiac catheterization, Holter monitoring, phonocardiography, stress testing, and vectorcardiography. The tests also detect and diagnose medical illnesses such as coronary artery disease, angina, arrhythmias and pericarditis.. The electrocardiograph technician is initially responsible for ...
I read the article by Yang JH,et al with great interest, in which the authors compared 1-year prognostic impacts of angiotensin receptor blockers (ARBs) with angiotensin converting enzyme inhibitors (ACEIs) in patients with ST segment elevation myocardial infarction (STEMI) with preserved left ventricular systolic function who underwent primary percutaneous coronary intervention (PCI) [1]. I believe it would be appreciated if authors discuss the long-term survival benefit of ARBs before concluding that ARBs are as beneficial as ACEIs in STEMI patients with preserved left ventricular systolic function after PCI.. Although ARBs could be an alternative to ACEIs, a recent observational study using inverse probability of treatment weighting and propensity score matching methods revealed that patients treated with ACEIs had significantly lower long-term mortality compared with those treated with ARBs from 2 to 5 years after acute myocardial infarction [2]. This study also demonstrated that crude ...
Of the 3250 patients, 634 sufferers without visual impairment were matched with 634 individuals with visual impairment. In the propensity score-matched evaluation, patients with visual impairment had a significantly higher threat of all-cause mortality compared with individuals without visual impairment in crude model (HR 1.72, 95 CI, 1.21?.45, P ?0.003), model 1 (HR 1.71, 95 CI, 1.21?.44, P ?0.003) and model two (HR 1.69, 95 CI, 1.12?.54, P ?0.01) even soon after adjusting for sex, DM, cardiovascular disease, health insurance, education, duration of dialysis, the use of ACEi or ARB, left ventricular hypertrophy on electrocardiogram, serum creatinine, serum albumin, and HbA1c.Subgroup Analysis of All-Cause Mortality by Risk Variables According to Visual ImpairmentSubgroup analysis associations involving visual impairment and all-cause mortality in many subgroups of individuals are displayed in Figure two. In subgroup analyses, there have been no substantial interactions amongst visual impairment ...
Right bundle branch block Differential diagnosis of right bundle branch block / causes of right bundle branch block are : -pulmonary embolism
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[92 Pages Report] Check for Discount on Global and China Diagnostic Electrocardiograph (ECG) Market Research by Company, Type & Application 2013-2025 report by HeyReport. Summary Diagnostic Electrocardiograph (ECG) is the process of recording the...
On- vs. off-hours admission of patients with ST-elevation acute myocardial infarction undergoing percutaneous coronary interventions: data from a tertiary university brazilian hospital
article{11bc16ce-648f-4e73-acd6-27c5bab30fc3, author = {Hedén, Bo and Ohlsson, Mattias and Holst, Holger and Mjöman, Mattias and Rittner, Ralf and Pahlm, Olle and Peterson, Carsten and Edenbrandt, Lars}, issn = {0002-9149}, language = {eng}, number = {5}, pages = {600--604}, publisher = {Excerpta Medica}, series = {American Journal of Cardiology}, title = {Detection of frequently overlooked electrocardiographic lead reversals using artificial neural networks}, volume = {78}, year = {1996 ...
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Indirect evidence of independent atrial activity Capture beat Occasionally an atrial impulse may cause ventricular depolarisation via the normal conduction system. The resulting QRS complex occurs earlier than expected and is narrow. indd 25 conduct normally, thus making a diagnosis of supraventricular tachycardia with aberrancy unlikely. Capture beats are uncommon, and though they confirm a diagnosis of ventricular tachycardia, their absence does not exclude the diagnosis. 8 Capture beat. 9 Fusion beat. 14 s). Consequently, this arrhythmia is commonly misdiagnosed as a supraventricular tachycardia. The QRS complexes have a right bundle branch block pattern, often with a small Q wave rather than primary R wave in lead V1 and a deep S wave in lead V6. When the tachycardia originates from the posterior fascicle the frontal plane axis of the QRS complex is deviated to the left; when it originates from the anterior fascicle, right axis deviation is seen. Right ventricular outflow tract tachycardia ...
|p|The 12-lead surface electrocardiogram adjacent QTc dispersion, which is the maximum difference of corrected QT interval between two adjacent leads, is a simple method to determine regional variation in repolarization and refractoriness. The aim of this study is to evaluate adjacent QTc dispersion as a marker of susceptibility to ventricular arrhythmias after myocardial infarction. A total of 135 consecutive patients with acute myocardial infarction were enrolled in the study. Adjacent QTc, measured by lens magnifier, was calculated on the first, second and third days after acute myocardial infarction. On the second day after acute myocardial infarction, adjacent QTc dispersion was significantly greater in patients with ventricular arrhythmias (P < 0.001). Adjacent QTc dispersion on the first and fifth day after acute myocardial infarction was not associated with development of ventricular arrhythmias. On the second day after acute myocardial infarction, adjacent QTc dispersion is
In 680 patients with acute myocardial infarction the prognosis during the following 5 years was related to observations made in a standard electrocardiogram (ECG) and 24 precordial chest leads. Patients with a Q-wave infarction (based on a 12-lead standard ECG) had a mortality rate during hospitalization of 10.2% which was much higher than that in patients with a non-Q-wave infarction (1.9%, p less than 0.001). At 5 years follow-up 33.6% of those with a Q-wave infarction had died versus 28.4% of those with a non-Q-wave infarction (p greater than 0.2). Corresponding mortality rate among patients with no previous infarction (n = 587) was 32.1% and 25.2%, respectively (p = 0.17). In patients with anterior infarction and no previous infarction there was no correlation between Q- and R-wave changes in the 24 chest leads 4 days after admission to hospital and 5-year mortality rate. We thus conclude that patients with a Q-wave infarction had a higher in-hospital mortality compared with non-Q-wave ...
INTRODUCTION: In the ECG, significant ST elevation or depression according to specific amplitude criteria can be indicative of acute coronary syndrome (ACS). Guidelines state that the ST amplitude should be measured at the J point, but data to support that this is the optimal measuring point for ACS detection is lacking. We evaluated the impact of different measuring points for ST deviation on the diagnostic accuracy for ACS in unselected emergency department (ED) chest pain patients.. MATERIAL AND METHODS: We included 14,148 adult patients with acute chest pain and an ECG recorded at a Swedish ED between 2010 and 2014. ST deviation was measured at the J point (STJ) and at 20, 40, 60 and 80 ms after the J point. A discharge diagnosis of ACS or not at the index visit was noted in all patients.. RESULTS: In total, 1489 (10.5%) patients had ACS. ST amplitude criteria at STJ had a sensitivity of 28% and a specificity of 92% for ACS. With these criteria, the highest positive and negative predictive ...
The relationship between R-wave amplitude and left ventricular volume was examined using two groups of patients, undergoing diagnostic cardiac catheterisation for investigation of chest pain, who had simultaneous R-wave recording and left ventricular angiography. R-wave amplitude was measured in leads 1, 2, 3 and V4-6. Left ventricular volume was altered by nitroglycerine (n = 18) and atrial pacing (n = 13). In both groups, increase or decrease in left ventricular volume was associated with a concomitant change of R-wave amplitude. We conclude that left ventricular volume is an important determinant of surface-recorded R waves with increased amplitude reflecting increased left ventricular volume and vice versa.
Symptom-to-door time in ST segment elevation myocardial infarction: overemphasized or overlooked? Results from the AMI-McGill study. Can J Cardiol. 2008 Mar; 24(3):213-6 ...
Aims: Left bundle branch block (LBBB) is generally associated with a poorer prognosis in comparison to normal intraventricular conduction, but also in comparison to right bundle branch block which is generally considered to be benign in the absence of an underlying cardiac disorder like congenital heart disease. In this paper we evaluate the presence of possible cardiovascular pathology in a group of nurses with a low level of cardiovascular risk factors and left bundle branch block (LBBB). Methods: During the period 2009-2013, 356 nurses (mean age: 32.6 ± 11 yr) were admitted to the department of Occupational Medicine of Second University of Naples. Of these, 13 had LBBB. The evaluation of these patients has included an electrocardiogram (ECG), echocardiography, 24-h ambulatory Holter monitoring (ECG Holter), and exercise testing. Subsequently, in patients with LBBB, multislice computed coronary angiography (MSCT) has been considered. Results and Conclusion: Only in one patient we found a
Paroxysmal ventricular tachycardia is most often associated with organic heart disease and in this setting is of ominous prognostic import (1-3). Less commonly, this arrhythmia occurs in young individuals without underlying heart disease; in these patients the disorder is generally considered benign (4). However, in some of these patients the episodes of paroxysmal ventricular tachycardia may lead to syncope and sudden death (2, 5). Recently, the efficacy of the beta-adrenergic blocking drugs in the treatment of tachyarrhythmias has been recognized (6, 7), and these drugs have been employed with success in certain patients with recurrent ventricular tachycardia (8-11). Electrical pacemakers ...
Congenital long QT syndrome (LQTS) is a genetic channelopathy associated with a high incidence of sudden cardiac death in children and young adults. QT interval prolongation is typically the primary finding on the electrocardiography (ECG) recordings, but a normal QT interval may be seen in as many as 40% of patients with LQTS due to incomplete penetrance. A normal QT interval on ECG in patients with LQTS is known as hidden LQTS. An epinephrine provocation test can help in the diagnosis of hidden LQTS. This case report describes the use of an epinephrine provocation test to diagnose hidden LQTS in 3 patients who had normal QT interval and corrected QT interval on ECG and a family history of sudden cardiac death. ...
Cardiology for Finals FY1s Poornima Mohan & Ghazal Saadat Overview • • • • • • Scars Acute coronary syndromes Valvular heart disease Infective Endocarditis Dextrocardia Arrhythmias Midline sternotomy scar What is this scar? Which 3 procedures would cause this scar? What else would you look for? Grafts What could this be? What are the indications? Where else should you look? We have this patient with chest pain 66 year old with a background of DM type 2, hypertension and a 40 pack yr smoking hx. Day 1 post inguinal hernia repair. Has been having central crushing chest pain for last 15 minutes. No relief from GTN. Hot & sweaty, vomited twice. Obs: BP- 120/60 P-75 RR- 24 Sats 98% on RA What ECG features suggest an STEMI?? ST elevation in 2mm in 2 or more contigous limb leads ST elevation in 2 or more contigous chest leads New onset LBBB Posterior MI . What features suggest an to NSTEMI ??? ST depression and /or T wave inversion in 2 or more leads. Risk is assessed using the TIMI ...
In this article, we will discuss the Diagnosis of Ventricular Tachycardia. So, lets get started.. Diagnosis. The ECG helps in the diagnosis. Wide QRS complexes (>0.14 seconds), at a regular rate of >100 bpm with presence of AV dissociation (independent P-wave not related to wide QRS complexes), concordant pattern, superior QRS axis, capture beats and fusion complexes favour the diagnosis of VT. It may be monomorphic (all QRS complexes alike originating from a single focus) or polymorphic (QRS complexes are not alike suggests organ from multiple foci). Torsades de pointee is a form of polymorphic VT. It must be stressed here that in spite of all these criteria, the ECG diagnosis of VT is not only difficult but may be impossible to differentiate it from PSVT with aberrant conduction (another common cause of wide QRS tachycardia) because there is no single electrocardiographic sign which confirms the diagnosis of VT.. Following are the common causes of wide QRS tachycardia:. ...
Results: LVH was diagnosed in 17 (47%) patients (6 women and 11 men). Following ECG parameters correlated the most prominently with LVMI - RV5: r = 0.5 (p = 0.002), RV6: r = 0.61 (p = 0.0001), SV1+RV5, 6: r = 0.64 (p = 0.001), RaVL+SV3: r = 0.5 (p = 0.002), SV2+RV5, 6: r = 0.71 (p = 0.0001), SV2, 3+RV5, 6: r = 0.75 (p = 0.0001). Based on the results of ROC analysis we proposed new cut points for LVH parameters. The highest diagnostic accuracy achieved S2+SV3 > 6 mV, SV2,V3+RV5,V6 > 4 mV, RaVL+SV3 > 3.5 mV (86-89 ...
The clinical and ventricular wall motion abnormality features of takotsubo cardiomyopathy are well defined. However, the underlying pathophysiology of this disorder is not completely understood. It has been suggested that takotsubo is a clinical syndrome with a multitude of predisposing factors, triggers, and pathogenic mechanisms, whose common final outcome is transient left ventricular systolic dysfunction characterized by apical ballooning with relative sparing of the basal segments. The syndrome is often preceded by acute stress (somatic and/or emotional) followed by chest pain, electrocardiographic abnormalities, and elevated cardiac troponin levels in the absence of obstructive coronary artery disease.. Massive catecholamine release and exaggerated sympathetic activation with elevated plasma catecholamines (up to 3-fold higher compared with patients presenting with acute myocardial infarction) are thought to play pivotal roles in the pathophysiology of takotsubo cardiomyopathy (1). ...
We recruited a random sample of men aged 50-69 years from the registers of general practices in the Merton, Sutton, and Wandsworth District Health Authority area, south London. A total of 612 men were invited and 413 (67%) attended. Of these, 25 were non-white and were excluded. Information was obtained on history and symptoms of coronary heart disease, lifestyle, and socioeconomic circumstances, as described previously. Cardiovascular risk factor profiles and serological tests for H pylori and C pneumoniae were also performed as described.11 Electrocardiograms were Minnesota coded. We took tracings to indicate coronary heart disease if they showed any of the following: Q waves, ST segment depression, left bundle branch block, or T wave inversion. Only the 303 men who had complete cardiovascular risk factor profiles were included in the study.. C Reactive protein concentration was measured by in house enzyme linked immunosorbent assay (ELISA). Rabbit antihuman C reactive protein (Dako) was used ...
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Left Bundle Branch Block Differential diagnosis of left bundle branch block / causes of left bundle branch block are : -ischemic heart disease
Brugada syndrome (BrS) is among the more common familial arrhythmia syndromes, with an estimated prevalence of 1 to 5 per 10 000 persons. It is characterized by a right ventricular conduction delay, dynamic or persistent ST-segment elevations in the precordial leads V1-3 , and an elevated risk of syncope and sudden cardiac death in young adults without structural heart disease... ...
... (ECG or EKG[a]) is the process of recording the electrical activity of the heart[4] over a period of time ... Electrocardiography Vectorcardiography. Holter monitor. Implantable loop recorder. Cardiac stress test Bruce protocol. ... Cooper JK (1986). "Electrocardiography 100 years ago. Origins, pioneers, and contributors". N Engl J Med. 315 (7): 461-64. doi: ... Pediatric and Fundamental Electrocardiography. Developments in Cardiovascular Medicine. 56. pp. 315-327. doi:10.1007/978-1-4613 ...
Electrocardiography[edit]. ECG pattern in Brugada syndrome. According to consensus guidelines, Type 1 ST segment elevation, ...
Retrieved from "https://en.wikipedia.org/w/index.php?title=P_wave_(electrocardiography)&oldid=842471189" ...
The presence of LBBB results in that electrocardiography (ECG) cannot be used to diagnose left ventricular hypertrophy or Q ... "Electrocardiography > Left Bundle Branch Block (LBBB)". Southern Illinois University School of Medicine. Retrieved 2015-07-02. ...
Conover, Mary Boudreau (2003). "Bedside Diagnosis". Understanding electrocardiography. St. Louis: Mosby. p. 82. ISBN 0-323- ...
Conover, Mary Boudreau (2003). Understanding Electrocardiography. Elsevier Health Sciences. pp. 117-118. ISBN 978-0323019057. ...
Einthoven's triangle is an imaginary formation of three limb leads in a triangle used in electrocardiography, formed by the two ... p. 5. ISBN 978-1-60547-476-2. Understanding Electrocardiography. Elsevier Health Sciences. 2003. p. 4. ISBN 0-323-01905-6. (in ... Snellen, H. A. (1995). Willem Einthoven (1860-1927), Father of Electrocardiography: Life and Work, Ancestors and Contemporaries ... Clinical Electrocardiography: A Textbook, p. 13 Lippincott Williams & Wilkins (1 August 2009). ECG Facts Made Incredibly Quick ...
Wenger, R; Massumi, RA; Kuramoto, K (1955). "A comparative study of esophageal and direct auricular electrocardiography in dog ... Fisch, Charles (1973). Complex electrocardiography. Philadelphia: F.A. Davis. ISBN 978-0-8036-3555-5. LCCN 73082285.[page ... "Complex Electrocardiography". He was a driving force behind the field's understanding of Prinzmetal-Massumi syndrome, a rare ... "A Comparative Study of Esophageal and Direct Auricular Electrocardiography in Dog". Cardiology. 26 (4): 193-208. doi:10.1159/ ...
... is an imaginary formation of three limb leads in a triangle used in electrocardiography, formed by the two ... p. 4. ISBN 0-323-01905-6. Silverman, M. E. (1992-10-01). "Willem Einthoven--the father of electrocardiography". Clinical ... p. 5. ISBN 978-1-60547-476-2. Understanding Electrocardiography. Elsevier Health Sciences. 2003. ...
Chugh, S. N. (2014-05-14). Textbook of Clinical Electrocardiography. Jaypee Brothers Publishers. ISBN 9789350906088. Burns, Ed ... Conover, Mary Boudreau (2003-01-01). Understanding Electrocardiography. Elsevier Health Sciences. ISBN 978-0323019057. Kossmann ...
Levine, Shel; Coyne, Brian J.; Colvin, Lisa Cooper (2015-02-13). Clinical Exercise Electrocardiography. Jones & Bartlett ...
"PSTF Paramedic Student Electrocardiography". Archived from the original on 2011-12-19. Retrieved 2010-03-27. Kenneth M Sutin; ...
Wilson in the field of electrocardiography. All of his work in this domain was done primarily in an effort to explain why ... Wilson's time in the last years of his active service was devoted to informal teaching of electrocardiography to doctors who ... Wilson's life was dedicated primarily to electrocardiography he had many other interests and hobbies. Both he and his wife ... Burch, George Edward; DePasquale, Nicholas P. (1990). A History of Electrocardiography. Norman Publishing. pp. 91-. ISBN ...
2002-03-02). "ABC of clinical electrocardiography". Retrieved 2008-04-28. v t e. ...
Besterman E, Creese R. (July 1979). "Waller-pioneer of electrocardiography". British Heart Journal. 42 (1): 61-64. doi:10.1136/ ...
Edhouse J, Thakur RK, Khalil JM (2002-05-25). "ABC of clinical electrocardiography. Conditions affecting the left side of the ... Edhouse, J; Thakur, RK; Khalil, JM (25 May 2002). "ABC of clinical electrocardiography. Conditions affecting the left side of ...
Besterman E, Creese R (July 1979). "Waller--pioneer of electrocardiography". Br Heart J. 42 (1): 61-64. doi:10.1136/hrt.42.1.61 ...
Baltazar, Romulo F. (2012-03-28). Basic and Bedside Electrocardiography. ISBN 9781451147919. Cleland JG, Daubert JC, Erdmann E ...
12-lead Holter electrocardiography. Review of the literature and clinical application update. Su L1, Borov S, Zrenner B. ... In medicine, a Holter monitor (often simply Holter) is a type of ambulatory electrocardiography device, a portable device for ... When used to study the heart, much like standard electrocardiography, the Holter monitor records electrical signals from the ... electrocardiography or ECG). Its extended recording period is sometimes useful for observing occasional cardiac arrhythmias ...
Electrocardiography 100 years ago. New England J Med1987;215:461-3] Bowbrick & Borg (2006), p. 10. 'Einthoven's String ... p. 2. 'Willem Einthoven and the Birth of Clinical Electrocardiography a Hundred Years Ago', S. Serge Barold, Cardiac ... He was a leader in applying the string galvanometer to physiology and medicine, leading to today's electrocardiography. ... and Thomas Lewis teamed to become the first of their profession to apply electrocardiography in medical diagnosis.[citation ...
Conover, Boudreau Conover (2003). Understanding Electrocardiography (8th ed.). St Louis: Mosby. p. 46. ISBN 9780323019057. ...
Electrocardiography; Use of cathode-ray oscilloscope to show magnitude and direction of galvanometer deflections caused by ... Cardiovascular Pressure Pulses and Electrocardiography; Case history (35 min., color). PMF 5213 - PMF 5214 - PMF 5215 - PMF ...
Evans, E. O.; Coigley, M. H.; Lewis, J. V.; Woodward, N. A. (October 1973). "Electrocardiography in general practice". The ...
Bernstine, Richard Lee (1961). Fetal electrocardiography and electroencephalography. Thomas. pp. 63-69. Urigüen, Jose Antonio; ...
Becker Daniel E (2006). "Fundamentals of Electrocardiography Interpretation". Anesthesia Progress. 53 (2): 53-64. doi:10.2344/ ... see electrocardiography for more details). Similar to skeletal muscle, the resting membrane potential (voltage when the cell is ...
Main article: Electrocardiography. Cardiac cycle shown against ECG. Using surface electrodes on the body, it is possible to ...
Shvilkin, Ary L. Goldberger, Zachary D. Goldberger, Alexei (2013). Goldberger's Clinical Electrocardiography: A Simplified ... Wagner, Galen S. (2001). Marriott's Practical Electrocardiography (10th ed.). Philadelphia, PA: Williams & Wilkins. ISBN ...
Snellen, Herman Adrianus (1995), Willem Einthoven (1860-1927) Father of Electrocardiography, Life and Work, Ancestors and ... This invention allowed transthoracic electrocardiography.[citation needed] Although later technological advances brought about ... Father of electrocardiography". Cardiology Journal. 14 (3): 316-317. PMID 18651479. Works by or about Willem Einthoven at ... Einthoven was awarded the Nobel Prize in Physiology or Medicine for inventing the first practical system of electrocardiography ...
Electrocardiography "Medical Definition of Vectorcardiography". www.merriam-webster.com. Retrieved June 8, 2017. Burch, G.E. ( ...
Clinical electrocardiography (1922) Clinical electrocardiograms; their interpretation and significance (1929) Cardiac clinics ( ...
Electrocardiography (ECG or EKG[a]) is the process of recording the electrical activity of the heart[4] over a period of time ... Electrocardiography Vectorcardiography. Holter monitor. Implantable loop recorder. Cardiac stress test Bruce protocol. ... Cooper JK (1986). "Electrocardiography 100 years ago. Origins, pioneers, and contributors". N Engl J Med. 315 (7): 461-64. doi: ... Pediatric and Fundamental Electrocardiography. Developments in Cardiovascular Medicine. 56. pp. 315-327. doi:10.1007/978-1-4613 ...
Retrieved from "https://en.wikipedia.org/w/index.php?title=P_wave_(electrocardiography)&oldid=842471189" ...
Fundamentals of electrocardiography interpretation. Anesth Prog. 2006;53:53-64.CrossRefPubMedPubMedCentralGoogle Scholar ... Miwa S., Mieszczanska H.Z. (2018) Cardiac Electrocardiography. In: Mieszczanska H., Budzikowski A. (eds) Cardiology Consult ... acute ischemia/infarction a scientific statement from the American Heart Association Electrocardiography and Arrhythmias ... associated with cardiac chamber hypertrophy a scientific statement from the American Heart Association Electrocardiography and ...
encoded search term (What is electrocardiography (ECG)?) and What is electrocardiography (ECG)? What to Read Next on Medscape. ... What is electrocardiography (ECG)?. Updated: Mar 11, 2019 * Author: Tarek Ajam, MD, MS; Chief Editor: Terrence X OBrien, MD, ... electrocardiography diagnostic statement list a scientific statement from the American Heart Association Electrocardiography ... ABC of clinical electrocardiography. Conditions affecting the left side of the heart. BMJ. 2002 May 25. 324 (7348):1264-7. [ ...
UNIPOLAR ELECTROCARDIOGRAPHY. Br Med J 1958; 1 doi: https://doi.org/10.1136/bmj.1.5084.1399-a (Published 14 June 1958) Cite ...
Electrocardiography with Music. Br Med J 1964; 2 doi: https://doi.org/10.1136/bmj.2.5422.1464-c (Published 05 December 1964) ...
Electrocardiography, method of tracing the electric current of a heartbeat to provide information on the heart. ... Electrocardiography, method of graphic tracing (electrocardiogram; ECG or EKG) of the electric current generated by the heart ... In addition, electrocardiography can be used to determine whether a slow heart rate is physiological or is caused by heart ... During the late 1960s, computerized electrocardiography came into use in many of the larger hospitals. ...
An improved electrocardiography system displays and records electrocardiograph (ECG) signals provided on multiple pickup leads ... Typically, in such electrocardiography systems, a large number of electrode leads are connected to the patient, but only a few ... 8. An electrocardiography system for displaying, in accordance with an operator input provided via control console switches, ... An electrocardiography system for displaying, in accordance with an operator input provided via control console switches, ...
This book deals with the basic concepts of high resolution electrocardiography: the electrophysiological basis of late ... arrhythmia electrocardiogram (ECG) electrocardiography electrophysiology myocardial infarction physiology Editors and ... 2.Section of Electrocardiography and ElectrophysiologyMount Sinai Medical CenterNew YorkUSA ... This book deals with the basic concepts of high resolution electrocardiography: the electrophysiological basis of late ...
... add an essential health care service to your repertoire with knowledge of electrocardiography.. ...
Electrocardiography PR interval QRS complex QT interval ST segment T wave U wave Longmore, Murray (2004). Oxford Handbook of ...
... set purposes have been achieved by providing a device allowing easy and quick positioning of electrodes for Electrocardiography ...
Electrocardiography for the anaesthetist by W. N. Rollason; 4 editions; First published in 1964; Subjects: Anesthesia, ... Are you sure you want to remove Electrocardiography for the anaesthetist from your list? ...
Spatial Vector Electrocardiography. The Clinical Characteristics of S-T and T Vectors. ROBERT P. GRANT, E. HARVEY ESTES, JOSEPH ...
Implications for Pre-Participation Cardiovascular Evaluation Using Electrocardiography Nabeel Sheikh, Michael Papadakis, Aneil ...
Electrocardiography in suspected myocardial infarction has the main purpose of detecting ischemia or acute coronary injury in ...
Make research projects and school reports about electrocardiography easy with credible articles from our FREE, online ... Electrocardiography Gale Encyclopedia of Medicine, 3rd ed. COPYRIGHT 2006 Thomson Gale. Electrocardiography. Definition. ... Electrocardiography Encyclopedia of Public Health COPYRIGHT 2002 The Gale Group Inc.. ELECTROCARDIOGRAPHY. The ... Electrocardiography. Definition. Electrocardiography is a commonly used, noninvasive procedure for recording electrical changes ...
Electrocardiography is a key investigation for patients with suspected cardiac disorders. Electrocardiograms (ECGs) provide the ...
Electrocardiography Basics is a new infographic science illustration poster design by Eleanor Lutz at TabletopWhale.com for ... Source: http://www.coolinfographics.com/blog/2016/10/20/electrocardiography-basics.html ...
Maximal Treadmill Exercise Electrocardiography. Correlations with Coronary Arteriography and Cardiac Hemodynamics. CARROLL M. ...
To more accurately trigger cardiac computed tomography angiography (CTA) than electrocardiography (ECG) alone, a sub-system is ...
Electrocardiography. Electrocardiographic (ECG) findings are abnormal in postpericardiotomy syndrome and may include the ...
Electrocardiography. ECG findings include prolonged QTc interval, elevated ST segments, and inverted T waves. [14] Although ...
CARD 3330 - Electrocardiography and Pacemakers. Cardiology/Cardiovascular Part-time Studies Course. School of Health Sciences. ... Interested in being notified about future offerings of CARD 3330 - Electrocardiography and Pacemakers? If so, fill out the ...
Explore electrocardiography profile at Times of India for photos, videos and latest news of electrocardiography. Also find news ... electrocardiography News: Latest and Breaking News on electrocardiography. ... Kumaraswamy N E often found himself irked by the blurred readings on the electrocardiography (ECG) machine. ...
Cardiovascular Disease Risk: Screening With Electrocardiography Description of Resource: The U.S. Preventive Services Task ... Home » Tools & Resources » Evidence-Based Resources » Cardiovascular Disease Risk: Screening With Electrocardiography ... Cardiovascular Disease Risk: Screening With Electrocardiography: Recommendation statement [Internet]. 2018 Jun [cited 2018 Jun ... Force (USPSTF) recommends against screening with resting or exercise electrocardiography (ECG) to prevent cardiovascular ...
View low-priced textbook options for Clinical Electrocardiography : A Simplified Approach on the eCampus.com Marketplace from ...
... embedded into an electrocardiography monitor (12), which correlates syncope events and electrocardiographic data. Physiological ... an abnormal electrocardiography baseline module configured to detect (242) whether an abnormal electrocardiography baseline is ... an abnormal electrocardiography baseline module configured to detect (242) whether an abnormal electrocardiography baseline is ... electrocardiography monitor 14. Other types of battery compartment are possible. The battery contained within the battery ...
Purchase Goldbergers Clinical Electrocardiography-A Simplified Approach: First South Asia Edition - 1st Edition. Print Book. ... Goldbergers Clinical Electrocardiography-A Simplified Approach: First South Asia Edition 1st Edition. ... Ideal for students and as a review for practicing clinicians, Goldbergers Clinical Electrocardiography explains the ... Associate Professor of Medicine, University of Washington School of Medicine, Director, Electrocardiography and Arrhythmia ...
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