Social structure of a group as it relates to the relative social rank of dominance status of its members. (APA, Thesaurus of Psychological Index Terms, 8th ed.)
The functional superiority and preferential use of one eye over the other. The term is usually applied to superiority in sighting (VISUAL PERCEPTION) or motor task but not difference in VISUAL ACUITY or dysfunction of one of the eyes. Ocular dominance can be modified by visual input and NEUROTROPHIC FACTORS.
The arterial blood vessels supplying the CEREBRUM.
Dominance of one cerebral hemisphere over the other in cerebral functions.
The formation of an area of NECROSIS in the CEREBRUM caused by an insufficiency of arterial or venous blood flow. Infarcts of the cerebrum are generally classified by hemisphere (i.e., left vs. right), lobe (e.g., frontal lobe infarction), arterial distribution (e.g., INFARCTION, ANTERIOR CEREBRAL ARTERY), and etiology (e.g., embolic infarction).
A heterogeneous group of nonprogressive motor disorders caused by chronic brain injuries that originate in the prenatal period, perinatal period, or first few years of life. The four major subtypes are spastic, athetoid, ataxic, and mixed cerebral palsy, with spastic forms being the most common. The motor disorder may range from difficulties with fine motor control to severe spasticity (see MUSCLE SPASTICITY) in all limbs. Spastic diplegia (Little disease) is the most common subtype, and is characterized by spasticity that is more prominent in the legs than in the arms. Pathologically, this condition may be associated with LEUKOMALACIA, PERIVENTRICULAR. (From Dev Med Child Neurol 1998 Aug;40(8):520-7)
The circulation of blood through the BLOOD VESSELS of the BRAIN.
The largest of the cerebral arteries. It trifurcates into temporal, frontal, and parietal branches supplying blood to most of the parenchyma of these lobes in the CEREBRAL CORTEX. These are the areas involved in motor, sensory, and speech activities.
The part of CENTRAL NERVOUS SYSTEM that is contained within the skull (CRANIUM). Arising from the NEURAL TUBE, the embryonic brain is comprised of three major parts including PROSENCEPHALON (the forebrain); MESENCEPHALON (the midbrain); and RHOMBENCEPHALON (the hindbrain). The developed brain consists of CEREBRUM; CEREBELLUM; and other structures in the BRAIN STEM.
Radiography of the vascular system of the brain after injection of a contrast medium.
NECROSIS occurring in the MIDDLE CEREBRAL ARTERY distribution system which brings blood to the entire lateral aspects of each CEREBRAL HEMISPHERE. Clinical signs include impaired cognition; APHASIA; AGRAPHIA; weak and numbness in the face and arms, contralaterally or bilaterally depending on the infarction.
A condition characterized by somnolence or coma in the presence of an acute infection with PLASMODIUM FALCIPARUM (and rarely other Plasmodium species). Initial clinical manifestations include HEADACHES; SEIZURES; and alterations of mentation followed by a rapid progression to COMA. Pathologic features include cerebral capillaries filled with parasitized erythrocytes and multiple small foci of cortical and subcortical necrosis. (From Adams et al., Principles of Neurology, 6th ed, p136)
Veins draining the cerebrum.
Localized reduction of blood flow to brain tissue due to arterial obstruction or systemic hypoperfusion. This frequently occurs in conjunction with brain hypoxia (HYPOXIA, BRAIN). Prolonged ischemia is associated with BRAIN INFARCTION.
Bleeding into one or both CEREBRAL HEMISPHERES including the BASAL GANGLIA and the CEREBRAL CORTEX. It is often associated with HYPERTENSION and CRANIOCEREBRAL TRAUMA.
Genes that influence the PHENOTYPE both in the homozygous and the heterozygous state.
Behavioral manifestations of cerebral dominance in which there is preferential use and superior functioning of either the left or the right side, as in the preferred use of the right hand or right foot.
Relationship between individuals when one individual threatens or becomes aggressive and the other individual remains passive or attempts to escape.
Images seen by one eye.
Any behavior associated with conflict between two individuals.
Non-invasive method of demonstrating internal anatomy based on the principle that atomic nuclei in a strong magnetic field absorb pulses of radiofrequency energy and emit them as radiowaves which can be reconstructed into computerized images. The concept includes proton spin tomographic techniques.
Brief reversible episodes of focal, nonconvulsive ischemic dysfunction of the brain having a duration of less than 24 hours, and usually less than one hour, caused by transient thrombotic or embolic blood vessel occlusion or stenosis. Events may be classified by arterial distribution, temporal pattern, or etiology (e.g., embolic vs. thrombotic). (From Adams et al., Principles of Neurology, 6th ed, pp814-6)
Area of the OCCIPITAL LOBE concerned with the processing of visual information relayed via VISUAL PATHWAYS.
The blending of separate images seen by each eye into one composite image.
Artery formed by the bifurcation of the internal carotid artery (CAROTID ARTERY, INTERNAL). Branches of the anterior cerebral artery supply the CAUDATE NUCLEUS; INTERNAL CAPSULE; PUTAMEN; SEPTAL NUCLEI; GYRUS CINGULI; and surfaces of the FRONTAL LOBE and PARIETAL LOBE.
A heterogeneous group of sporadic or familial disorders characterized by AMYLOID deposits in the walls of small and medium sized blood vessels of CEREBRAL CORTEX and MENINGES. Clinical features include multiple, small lobar CEREBRAL HEMORRHAGE; cerebral ischemia (BRAIN ISCHEMIA); and CEREBRAL INFARCTION. Cerebral amyloid angiopathy is unrelated to generalized AMYLOIDOSIS. Amyloidogenic peptides in this condition are nearly always the same ones found in ALZHEIMER DISEASE. (from Kumar: Robbins and Cotran: Pathologic Basis of Disease, 7th ed., 2005)
Artery formed by the bifurcation of the BASILAR ARTERY. Branches of the posterior cerebral artery supply portions of the OCCIPITAL LOBE; PARIETAL LOBE; inferior temporal gyrus, brainstem, and CHOROID PLEXUS.
The absence or restriction of the usual external sensory stimuli to which the individual responds.
Four CSF-filled (see CEREBROSPINAL FLUID) cavities within the cerebral hemispheres (LATERAL VENTRICLES), in the midline (THIRD VENTRICLE) and within the PONS and MEDULLA OBLONGATA (FOURTH VENTRICLE).
Processes and properties of the EYE as a whole or of any of its parts.
Increased intracellular or extracellular fluid in brain tissue. Cytotoxic brain edema (swelling due to increased intracellular fluid) is indicative of a disturbance in cell metabolism, and is commonly associated with hypoxic or ischemic injuries (see HYPOXIA, BRAIN). An increase in extracellular fluid may be caused by increased brain capillary permeability (vasogenic edema), an osmotic gradient, local blockages in interstitial fluid pathways, or by obstruction of CSF flow (e.g., obstructive HYDROCEPHALUS). (From Childs Nerv Syst 1992 Sep; 8(6):301-6)
Bleeding into the intracranial or spinal SUBARACHNOID SPACE, most resulting from INTRACRANIAL ANEURYSM rupture. It can occur after traumatic injuries (SUBARACHNOID HEMORRHAGE, TRAUMATIC). Clinical features include HEADACHE; NAUSEA; VOMITING, nuchal rigidity, variable neurological deficits and reduced mental status.
Elements of limited time intervals, contributing to particular results or situations.
Derived from TELENCEPHALON, cerebrum is composed of a right and a left hemisphere. Each contains an outer cerebral cortex and a subcortical basal ganglia. The cerebrum includes all parts within the skull except the MEDULLA OBLONGATA, the PONS, and the CEREBELLUM. Cerebral functions include sensorimotor, emotional, and intellectual activities.
A spectrum of pathological conditions of impaired blood flow in the brain. They can involve vessels (ARTERIES or VEINS) in the CEREBRUM, the CEREBELLUM, and the BRAIN STEM. Major categories include INTRACRANIAL ARTERIOVENOUS MALFORMATIONS; BRAIN ISCHEMIA; CEREBRAL HEMORRHAGE; and others.
A value equal to the total volume flow divided by the cross-sectional area of the vascular bed.
Embolism or thrombosis involving blood vessels which supply intracranial structures. Emboli may originate from extracranial or intracranial sources. Thrombosis may occur in arterial or venous structures.
The domestic cat, Felis catus, of the carnivore family FELIDAE, comprising over 30 different breeds. The domestic cat is descended primarily from the wild cat of Africa and extreme southwestern Asia. Though probably present in towns in Palestine as long ago as 7000 years, actual domestication occurred in Egypt about 4000 years ago. (From Walker's Mammals of the World, 6th ed, p801)
Social rank-order established by certain behavioral patterns.
Pathologic conditions affecting the BRAIN, which is composed of the intracranial components of the CENTRAL NERVOUS SYSTEM. This includes (but is not limited to) the CEREBRAL CORTEX; intracranial white matter; BASAL GANGLIA; THALAMUS; HYPOTHALAMUS; BRAIN STEM; and CEREBELLUM.
Pressure within the cranial cavity. It is influenced by brain mass, the circulatory system, CSF dynamics, and skull rigidity.
Abnormal outpouching in the wall of intracranial blood vessels. Most common are the saccular (berry) aneurysms located at branch points in CIRCLE OF WILLIS at the base of the brain. Vessel rupture results in SUBARACHNOID HEMORRHAGE or INTRACRANIAL HEMORRHAGES. Giant aneurysms (>2.5 cm in diameter) may compress adjacent structures, including the OCULOMOTOR NERVE. (From Adams et al., Principles of Neurology, 6th ed, p841)
Microsurgical revascularization to improve intracranial circulation. It usually involves joining the extracranial circulation to the intracranial circulation but may include extracranial revascularization (e.g., subclavian-vertebral artery bypass, subclavian-external carotid artery bypass). It is performed by joining two arteries (direct anastomosis or use of graft) or by free autologous transplantation of highly vascularized tissue to the surface of the brain.
Constriction of arteries in the SKULL due to sudden, sharp, and often persistent smooth muscle contraction in blood vessels. Intracranial vasospasm results in reduced vessel lumen caliber, restricted blood flow to the brain, and BRAIN ISCHEMIA that may lead to hypoxic-ischemic brain injury (HYPOXIA-ISCHEMIA, BRAIN).
Theoretical representations that simulate the behavior or activity of genetic processes or phenomena. They include the use of mathematical equations, computers, and other electronic equipment.
The adaptive superiority of the heterozygous GENOTYPE with respect to one or more characters in comparison with the corresponding HOMOZYGOTE.
The basic cellular units of nervous tissue. Each neuron consists of a body, an axon, and dendrites. Their purpose is to receive, conduct, and transmit impulses in the NERVOUS SYSTEM.
Deliberate breeding of two different individuals that results in offspring that carry part of the genetic material of each parent. The parent organisms must be genetically compatible and may be from different varieties or closely related species.
A reduction in brain oxygen supply due to ANOXEMIA (a reduced amount of oxygen being carried in the blood by HEMOGLOBIN), or to a restriction of the blood supply to the brain, or both. Severe hypoxia is referred to as anoxia, and is a relatively common cause of injury to the central nervous system. Prolonged brain anoxia may lead to BRAIN DEATH or a PERSISTENT VEGETATIVE STATE. Histologically, this condition is characterized by neuronal loss which is most prominent in the HIPPOCAMPUS; GLOBUS PALLIDUS; CEREBELLUM; and inferior olives.
A colorless, odorless gas that can be formed by the body and is necessary for the respiration cycle of plants and animals.
Naturally occurring or experimentally induced animal diseases with pathological processes sufficiently similar to those of human diseases. They are used as study models for human diseases.
Behavior which may be manifested by destructive and attacking action which is verbal or physical, by covert attitudes of hostility or by obstructionism.
An element with atomic symbol O, atomic number 8, and atomic weight [15.99903; 15.99977]. It is the most abundant element on earth and essential for respiration.
Drugs intended to prevent damage to the brain or spinal cord from ischemia, stroke, convulsions, or trauma. Some must be administered before the event, but others may be effective for some time after. They act by a variety of mechanisms, but often directly or indirectly minimize the damage produced by endogenous excitatory amino acids.
A strain of albino rat used widely for experimental purposes because of its calmness and ease of handling. It was developed by the Sprague-Dawley Animal Company.
A specific stage in animal and human development during which certain types of behavior normally are shaped and molded for life.
Tomography using x-ray transmission and a computer algorithm to reconstruct the image.
The direct struggle between individuals for environmental necessities or for a common goal.
The mating of plants or non-human animals which are closely related genetically.
Unstable isotopes of xenon that decay or disintegrate emitting radiation. Xe atoms with atomic weights 121-123, 125, 127, 133, 135, 137-145 are radioactive xenon isotopes.
The artery formed by the union of the right and left vertebral arteries; it runs from the lower to the upper border of the pons, where it bifurcates into the two posterior cerebral arteries.
Imaging techniques used to colocalize sites of brain functions or physiological activity with brain structures.
The innermost layer of the three meninges covering the brain and spinal cord. It is the fine vascular membrane that lies under the ARACHNOID and the DURA MATER.
A group of pathological conditions characterized by sudden, non-convulsive loss of neurological function due to BRAIN ISCHEMIA or INTRACRANIAL HEMORRHAGES. Stroke is classified by the type of tissue NECROSIS, such as the anatomic location, vasculature involved, etiology, age of the affected individual, and hemorrhagic vs. non-hemorrhagic nature. (From Adams et al., Principles of Neurology, 6th ed, pp777-810)
Specialized non-fenestrated tightly-joined ENDOTHELIAL CELLS with TIGHT JUNCTIONS that form a transport barrier for certain substances between the cerebral capillaries and the BRAIN tissue.
A noninvasive technique that uses the differential absorption properties of hemoglobin and myoglobin to evaluate tissue oxygenation and indirectly can measure regional hemodynamics and blood flow. Near-infrared light (NIR) can propagate through tissues and at particular wavelengths is differentially absorbed by oxygenated vs. deoxygenated forms of hemoglobin and myoglobin. Illumination of intact tissue with NIR allows qualitative assessment of changes in the tissue concentration of these molecules. The analysis is also used to determine body composition.
PRESSURE of the BLOOD on the ARTERIES and other BLOOD VESSELS.
Restoration of blood supply to tissue which is ischemic due to decrease in normal blood supply. The decrease may result from any source including atherosclerotic obstruction, narrowing of the artery, or surgical clamping. It is primarily a procedure for treating infarction or other ischemia, by enabling viable ischemic tissue to recover, thus limiting further necrosis. However, it is thought that reperfusion can itself further damage the ischemic tissue, causing REPERFUSION INJURY.
Set of cell bodies and nerve fibers conducting impulses from the eyes to the cerebral cortex. It includes the RETINA; OPTIC NERVE; optic tract; and geniculocalcarine tract.
A form of gene interaction whereby the expression of one gene interferes with or masks the expression of a different gene or genes. Genes whose expression interferes with or masks the effects of other genes are said to be epistatic to the effected genes. Genes whose expression is affected (blocked or masked) are hypostatic to the interfering genes.
The flow of BLOOD through or around an organ or region of the body.
Tomography using radioactive emissions from injected RADIONUCLIDES and computer ALGORITHMS to reconstruct an image.
Recording of electric currents developed in the brain by means of electrodes applied to the scalp, to the surface of the brain, or placed within the substance of the brain.
The different ways GENES and their ALLELES interact during the transmission of genetic traits that effect the outcome of GENE EXPRESSION.
The difference between two images on the retina when looking at a visual stimulus. This occurs since the two retinas do not have the same view of the stimulus because of the location of our eyes. Thus the left eye does not get exactly the same view as the right eye.
A statistical technique that isolates and assesses the contributions of categorical independent variables to variation in the mean of a continuous dependent variable.
Investigative technique commonly used during ELECTROENCEPHALOGRAPHY in which a series of bright light flashes or visual patterns are used to elicit brain activity.
The capacity of the NERVOUS SYSTEM to change its reactivity as the result of successive activations.
Volume of circulating BLOOD. It is the sum of the PLASMA VOLUME and ERYTHROCYTE VOLUME.
Non-invasive method of vascular imaging and determination of internal anatomy without injection of contrast media or radiation exposure. The technique is used especially in CEREBRAL ANGIOGRAPHY as well as for studies of other vascular structures.
The observable response an animal makes to any situation.
Formation or presence of a blood clot (THROMBUS) in a blood vessel within the SKULL. Intracranial thrombosis can lead to thrombotic occlusions and BRAIN INFARCTION. The majority of the thrombotic occlusions are associated with ATHEROSCLEROSIS.
Tissue NECROSIS in any area of the brain, including the CEREBRAL HEMISPHERES, the CEREBELLUM, and the BRAIN STEM. Brain infarction is the result of a cascade of events initiated by inadequate blood flow through the brain that is followed by HYPOXIA and HYPOGLYCEMIA in brain tissue. Damage may be temporary, permanent, selective or pan-necrosis.
A technique of inputting two-dimensional images into a computer and then enhancing or analyzing the imagery into a form that is more useful to the human observer.
A gamma-emitting RADIONUCLIDE IMAGING agent used in the evaluation of regional cerebral blood flow and in non-invasive dynamic biodistribution studies and MYOCARDIAL PERFUSION IMAGING. It has also been used to label leukocytes in the investigation of INFLAMMATORY BOWEL DISEASES.
Congenital vascular anomalies in the brain characterized by direct communication between an artery and a vein without passing through the CAPILLARIES. The locations and size of the shunts determine the symptoms including HEADACHES; SEIZURES; STROKE; INTRACRANIAL HEMORRHAGES; mass effect; and vascular steal effect.
A carbamate insecticide.
Pathological processes which result in the partial or complete obstruction of ARTERIES. They are characterized by greatly reduced or absence of blood flow through these vessels. They are also known as arterial insufficiency.
One of the CARBONIC ANHYDRASE INHIBITORS that is sometimes effective against absence seizures. It is sometimes useful also as an adjunct in the treatment of tonic-clonic, myoclonic, and atonic seizures, particularly in women whose seizures occur or are exacerbated at specific times in the menstrual cycle. However, its usefulness is transient often because of rapid development of tolerance. Its antiepileptic effect may be due to its inhibitory effect on brain carbonic anhydrase, which leads to an increased transneuronal chloride gradient, increased chloride current, and increased inhibition. (From Smith and Reynard, Textbook of Pharmacology, 1991, p337)
The genetic process of crossbreeding between genetically dissimilar parents to produce a hybrid.
The outward appearance of the individual. It is the product of interactions between genes, and between the GENOTYPE and the environment.
The selecting and organizing of visual stimuli based on the individual's past experience.
Changes in the amounts of various chemicals (neurotransmitters, receptors, enzymes, and other metabolites) specific to the area of the central nervous system contained within the head. These are monitored over time, during sensory stimulation, or under different disease states.
Genotypic differences observed among individuals in a population.
A polygonal anastomosis at the base of the brain formed by the internal carotid (CAROTID ARTERY, INTERNAL), proximal parts of the anterior, middle, and posterior cerebral arteries (ANTERIOR CEREBRAL ARTERY; MIDDLE CEREBRAL ARTERY; POSTERIOR CEREBRAL ARTERY), the anterior communicating artery and the posterior communicating arteries.
A strain of albino rat developed at the Wistar Institute that has spread widely at other institutions. This has markedly diluted the original strain.
Any behavior caused by or affecting another individual, usually of the same species.
A family of large terrestrial carnivores possessing long legs, coarse guard hairs and a busy tail. It is comprised of hyenas and aardwolves.
The making of a radiograph of an object or tissue by recording on a photographic plate the radiation emitted by radioactive material within the object. (Dorland, 27th ed)
Infections of the BRAIN caused by the protozoan TOXOPLASMA gondii that primarily arise in individuals with IMMUNOLOGIC DEFICIENCY SYNDROMES (see also AIDS-RELATED OPPORTUNISTIC INFECTIONS). The infection may involve the brain diffusely or form discrete abscesses. Clinical manifestations include SEIZURES, altered mentation, headache, focal neurologic deficits, and INTRACRANIAL HYPERTENSION. (From Joynt, Clinical Neurology, 1998, Ch27, pp41-3)
Acute and chronic (see also BRAIN INJURIES, CHRONIC) injuries to the brain, including the cerebral hemispheres, CEREBELLUM, and BRAIN STEM. Clinical manifestations depend on the nature of injury. Diffuse trauma to the brain is frequently associated with DIFFUSE AXONAL INJURY or COMA, POST-TRAUMATIC. Localized injuries may be associated with NEUROBEHAVIORAL MANIFESTATIONS; HEMIPARESIS, or other focal neurologic deficits.
The total process by which organisms produce offspring. (Stedman, 25th ed)
A noble gas with the atomic symbol Xe, atomic number 54, and atomic weight 131.30. It is found in the earth's atmosphere and has been used as an anesthetic.
A genus of the subfamily CERCOPITHECINAE, family CERCOPITHECIDAE, consisting of five named species: PAPIO URSINUS (chacma baboon), PAPIO CYNOCEPHALUS (yellow baboon), PAPIO PAPIO (western baboon), PAPIO ANUBIS (or olive baboon), and PAPIO HAMADRYAS (hamadryas baboon). Members of the Papio genus inhabit open woodland, savannahs, grassland, and rocky hill country. Some authors consider MANDRILLUS a subgenus of Papio.
The gradual irreversible changes in structure and function of an organism that occur as a result of the passage of time.
Compounds that contain the radical R2C=N.OH derived from condensation of ALDEHYDES or KETONES with HYDROXYLAMINE. Members of this group are CHOLINESTERASE REACTIVATORS.
A condition of an inequality of refractive power of the two eyes.
A verbal or nonverbal means of communicating ideas or feelings.
Refers to animals in the period of time just after birth.
A clinical manifestation of abnormal increase in the amount of carbon dioxide in arterial blood.
Differential and non-random reproduction of different genotypes, operating to alter the gene frequencies within a population.
A method of computed tomography that uses radionuclides which emit a single photon of a given energy. The camera is rotated 180 or 360 degrees around the patient to capture images at multiple positions along the arc. The computer is then used to reconstruct the transaxial, sagittal, and coronal images from the 3-dimensional distribution of radionuclides in the organ. The advantages of SPECT are that it can be used to observe biochemical and physiological processes as well as size and volume of the organ. The disadvantage is that, unlike positron-emission tomography where the positron-electron annihilation results in the emission of 2 photons at 180 degrees from each other, SPECT requires physical collimation to line up the photons, which results in the loss of many available photons and hence degrades the image.
Variant forms of the same gene, occupying the same locus on homologous CHROMOSOMES, and governing the variants in production of the same gene product.
The processes whereby the internal environment of an organism tends to remain balanced and stable.
Assessment of sensory and motor responses and reflexes that is used to determine impairment of the nervous system.
Adverse functional, metabolic, or structural changes in ischemic tissues resulting from the restoration of blood flow to the tissue (REPERFUSION), including swelling; HEMORRHAGE; NECROSIS; and damage from FREE RADICALS. The most common instance is MYOCARDIAL REPERFUSION INJURY.
A genus of the subfamily CERCOPITHECINAE, family CERCOPITHECIDAE, consisting of 16 species inhabiting forests of Africa, Asia, and the islands of Borneo, Philippines, and Celebes.
Genetic loci associated with a QUANTITATIVE TRAIT.
The circulation of the BLOOD through the MICROVASCULAR NETWORK.
Part of the DIENCEPHALON inferior to the caudal end of the dorsal THALAMUS. Includes the lateral geniculate body which relays visual impulses from the OPTIC TRACT to the calcarine cortex, and the medial geniculate body which relays auditory impulses from the lateral lemniscus to the AUDITORY CORTEX.
An amphetamine analog that is rapidly taken up by the lungs and from there redistributed primarily to the brain and liver. It is used in brain radionuclide scanning with I-123.
The tearing or bursting of the weakened wall of the aneurysmal sac, usually heralded by sudden worsening pain. The great danger of a ruptured aneurysm is the large amount of blood spilling into the surrounding tissues and cavities, causing HEMORRHAGIC SHOCK.
Any detectable and heritable change in the genetic material that causes a change in the GENOTYPE and which is transmitted to daughter cells and to succeeding generations.
The chromosome region which is active in nucleolus formation and which functions in the synthesis of ribosomal RNA.
Formation or presence of a blood clot (THROMBUS) in the CRANIAL SINUSES, large endothelium-lined venous channels situated within the SKULL. Intracranial sinuses, also called cranial venous sinuses, include the superior sagittal, cavernous, lateral, petrous sinuses, and many others. Cranial sinus thrombosis can lead to severe HEADACHE; SEIZURE; and other neurological defects.
The rate at which oxygen is used by a tissue; microliters of oxygen STPD used per milligram of tissue per hour; the rate at which oxygen enters the blood from alveolar gas, equal in the steady state to the consumption of oxygen by tissue metabolism throughout the body. (Stedman, 25th ed, p346)
The production of offspring by selective mating or HYBRIDIZATION, GENETIC in animals or plants.
The range or frequency distribution of a measurement in a population (of organisms, organs or things) that has not been selected for the presence of disease or abnormality.
The smallest divisions of the arteries located between the muscular arteries and the capillaries.
The genetic constitution of the individual, comprising the ALLELES present at each GENETIC LOCUS.
Sexual activities of animals.
A functional system which includes the organisms of a natural community together with their environment. (McGraw Hill Dictionary of Scientific and Technical Terms, 4th ed)
Abnormally low BODY TEMPERATURE that is intentionally induced in warm-blooded animals by artificial means. In humans, mild or moderate hypothermia has been used to reduce tissue damages, particularly after cardiac or spinal cord injuries and during subsequent surgeries.
A calcium channel blockader with preferential cerebrovascular activity. It has marked cerebrovascular dilating effects and lowers blood pressure.
Pathological processes or diseases where cerebral MICROVESSELS show abnormalities. They are often associated with aging, hypertension and risk factors for lacunar infarcts (see LACUNAR INFARCTION); LEUKOARAIOSIS; and CEREBRAL HEMORRHAGE.
A subfamily of the Muridae consisting of several genera including Gerbillus, Rhombomys, Tatera, Meriones, and Psammomys.
A method of non-invasive, continuous measurement of MICROCIRCULATION. The technique is based on the values of the DOPPLER EFFECT of low-power laser light scattered randomly by static structures and moving tissue particulates.
Determination of the shortest time interval between the injection of a substance in the vein and its arrival at some distant site in sufficient concentration to produce a recognizable end result. It represents approximately the inverse of the average velocity of blood flow between two points.
Increased pressure within the cranial vault. This may result from several conditions, including HYDROCEPHALUS; BRAIN EDEMA; intracranial masses; severe systemic HYPERTENSION; PSEUDOTUMOR CEREBRI; and other disorders.
Severe or complete loss of motor function on one side of the body. This condition is usually caused by BRAIN DISEASES that are localized to the cerebral hemisphere opposite to the side of weakness. Less frequently, BRAIN STEM lesions; cervical SPINAL CORD DISEASES; PERIPHERAL NERVOUS SYSTEM DISEASES; and other conditions may manifest as hemiplegia. The term hemiparesis (see PARESIS) refers to mild to moderate weakness involving one side of the body.
A barbiturate with hypnotic and sedative properties (but not antianxiety). Adverse effects are mainly a consequence of dose-related CNS depression and the risk of dependence with continued use is high. (From Martindale, The Extra Pharmacopoeia, 30th ed, p565)
Evaluation undertaken to assess the results or consequences of management and procedures used in combating disease in order to determine the efficacy, effectiveness, safety, and practicability of these interventions in individual cases or series.
Paired bodies containing mostly GRAY MATTER and forming part of the lateral wall of the THIRD VENTRICLE of the brain.
NECROSIS occurring in the ANTERIOR CEREBRAL ARTERY system, including branches such as Heubner's artery. These arteries supply blood to the medial and superior parts of the CEREBRAL HEMISPHERE, Infarction in the anterior cerebral artery usually results in sensory and motor impairment in the lower body.
Narrowing or stricture of any part of the CAROTID ARTERIES, most often due to atherosclerotic plaque formation. Ulcerations may form in atherosclerotic plaques and induce THROMBUS formation. Platelet or cholesterol emboli may arise from stenotic carotid lesions and induce a TRANSIENT ISCHEMIC ATTACK; CEREBROVASCULAR ACCIDENT; or temporary blindness (AMAUROSIS FUGAX). (From Adams et al., Principles of Neurology, 6th ed, pp 822-3)
Clinical manifestation consisting of a deficiency of carbon dioxide in arterial blood.
Any operation on the cranium or incision into the cranium. (Dorland, 28th ed)
Unstable isotopes of oxygen that decay or disintegrate emitting radiation. O atoms with atomic weights 13, 14, 15, 19, and 20 are radioactive oxygen isotopes.
Either of the two principal arteries on both sides of the neck that supply blood to the head and neck; each divides into two branches, the internal carotid artery and the external carotid artery.
The process in which light signals are transformed by the PHOTORECEPTOR CELLS into electrical signals which can then be transmitted to the brain.
A disorder characterized by a reduction of oxygen in the blood combined with reduced blood flow (ISCHEMIA) to the brain from a localized obstruction of a cerebral artery or from systemic hypoperfusion. Prolonged hypoxia-ischemia is associated with ISCHEMIC ATTACK, TRANSIENT; BRAIN INFARCTION; BRAIN EDEMA; COMA; and other conditions.
The part of the cerebral hemisphere anterior to the central sulcus, and anterior and superior to the lateral sulcus.
The restriction of a characteristic behavior, anatomical structure or physical system, such as immune response; metabolic response, or gene or gene variant to the members of one species. It refers to that property which differentiates one species from another but it is also used for phylogenetic levels higher or lower than the species.
The pressure that would be exerted by one component of a mixture of gases if it were present alone in a container. (From McGraw-Hill Dictionary of Scientific and Technical Terms, 6th ed)
NECROSIS induced by ISCHEMIA in the POSTERIOR CEREBRAL ARTERY distribution system which supplies portions of the BRAIN STEM; the THALAMUS; TEMPORAL LOBE, and OCCIPITAL LOBE. Depending on the size and location of infarction, clinical features include OLFACTION DISORDERS and visual problems (AGNOSIA; ALEXIA; HEMIANOPSIA).
Semidomesticated variety of European polecat much used for hunting RODENTS and/or RABBITS and as a laboratory animal. It is in the subfamily Mustelinae, family MUSTELIDAE.
The movement and the forces involved in the movement of the blood through the CARDIOVASCULAR SYSTEM.
An analgesic and antipyretic that has been given by mouth and as ear drops. Antipyrine is often used in testing the effects of other drugs or diseases on drug-metabolizing enzymes in the liver. (From Martindale, The Extra Pharmacopoeia, 30th ed, p29)
Theoretical representations that simulate the behavior or activity of biological processes or diseases. For disease models in living animals, DISEASE MODELS, ANIMAL is available. Biological models include the use of mathematical equations, computers, and other electronic equipment.
A profound state of unconsciousness associated with depressed cerebral activity from which the individual cannot be aroused. Coma generally occurs when there is dysfunction or injury involving both cerebral hemispheres or the brain stem RETICULAR FORMATION.
The process of cumulative change over successive generations through which organisms acquire their distinguishing morphological and physiological characteristics.
Descriptions of specific amino acid, carbohydrate, or nucleotide sequences which have appeared in the published literature and/or are deposited in and maintained by databanks such as GENBANK, European Molecular Biology Laboratory (EMBL), National Biomedical Research Foundation (NBRF), or other sequence repositories.
A noninflammatory, progressive occlusion of the intracranial CAROTID ARTERIES and the formation of netlike collateral arteries arising from the CIRCLE OF WILLIS. Cerebral angiogram shows the puff-of-smoke (moyamoya) collaterals at the base of the brain. It is characterized by endothelial HYPERPLASIA and FIBROSIS with thickening of arterial walls. This disease primarily affects children but can also occur in adults.
Measurement of oxygen and carbon dioxide in the blood.
Observation of a population for a sufficient number of persons over a sufficient number of years to generate incidence or mortality rates subsequent to the selection of the study group.
Organic compounds that contain technetium as an integral part of the molecule. These compounds are often used as radionuclide imaging agents.
The variety of all native living organisms and their various forms and interrelationships.
Behavior in defense of an area against another individual or individuals primarily of the same species.
Genetically identical individuals developed from brother and sister matings which have been carried out for twenty or more generations or by parent x offspring matings carried out with certain restrictions. This also includes animals with a long history of closed colony breeding.
Histochemical localization of immunoreactive substances using labeled antibodies as reagents.
A characteristic showing quantitative inheritance such as SKIN PIGMENTATION in humans. (From A Dictionary of Genetics, 4th ed)
Age as a constituent element or influence contributing to the production of a result. It may be applicable to the cause or the effect of a circumstance. It is used with human or animal concepts but should be differentiated from AGING, a physiological process, and TIME FACTORS which refers only to the passage of time.
The coordination of a sensory or ideational (cognitive) process and a motor activity.
A diagnostic technique that incorporates the measurement of molecular diffusion (such as water or metabolites) for tissue assessment by MRI. The degree of molecular movement can be measured by changes of apparent diffusion coefficient (ADC) with time, as reflected by tissue microstructure. Diffusion MRI has been used to study BRAIN ISCHEMIA and tumor response to treatment.
The domestic dog, Canis familiaris, comprising about 400 breeds, of the carnivore family CANIDAE. They are worldwide in distribution and live in association with people. (Walker's Mammals of the World, 5th ed, p1065)
A condition characterized by long-standing brain dysfunction or damage, usually of three months duration or longer. Potential etiologies include BRAIN INFARCTION; certain NEURODEGENERATIVE DISORDERS; CRANIOCEREBRAL TRAUMA; ANOXIA, BRAIN; ENCEPHALITIS; certain NEUROTOXICITY SYNDROMES; metabolic disorders (see BRAIN DISEASES, METABOLIC); and other conditions.
The physiological narrowing of BLOOD VESSELS by contraction of the VASCULAR SMOOTH MUSCLE.
The physical measurements of a body.
A genus of the family CEBIDAE consisting of four species: S. boliviensis, S. orstedii (red-backed squirrel monkey), S. sciureus (common squirrel monkey), and S. ustus. They inhabit tropical rain forests in Central and South America. S. sciureus is used extensively in research studies.
Communication between animals involving the giving off by one individual of some chemical or physical signal, that, on being received by another, influences its behavior.
The statistical reproducibility of measurements (often in a clinical context), including the testing of instrumentation or techniques to obtain reproducible results. The concept includes reproducibility of physiological measurements, which may be used to develop rules to assess probability or prognosis, or response to a stimulus; reproducibility of occurrence of a condition; and reproducibility of experimental results.
Disease having a short and relatively severe course.
An individual having different alleles at one or more loci regarding a specific character.
An infant during the first month after birth.
The status during which female mammals carry their developing young (EMBRYOS or FETUSES) in utero before birth, beginning from FERTILIZATION to BIRTH.
Large endothelium-lined venous channels situated between the two layers of DURA MATER, the endosteal and the meningeal layers. They are devoid of valves and are parts of the venous system of dura mater. Major cranial sinuses include a postero-superior group (such as superior sagittal, inferior sagittal, straight, transverse, and occipital) and an antero-inferior group (such as cavernous, petrosal, and basilar plexus).
The part of brain that lies behind the BRAIN STEM in the posterior base of skull (CRANIAL FOSSA, POSTERIOR). It is also known as the "little brain" with convolutions similar to those of CEREBRAL CORTEX, inner white matter, and deep cerebellar nuclei. Its function is to coordinate voluntary movements, maintain balance, and learn motor skills.
A pulmonary ventilation rate faster than is metabolically necessary for the exchange of gases. It is the result of an increased frequency of breathing, an increased tidal volume, or a combination of both. It causes an excess intake of oxygen and the blowing off of carbon dioxide.
Number of individuals in a population relative to space.
A primary source of energy for living organisms. It is naturally occurring and is found in fruits and other parts of plants in its free state. It is used therapeutically in fluid and nutrient replacement.
Theoretical representations that simulate the behavior or activity of the neurological system, processes or phenomena; includes the use of mathematical equations, computers, and other electronic equipment.
Posterior portion of the CEREBRAL HEMISPHERES responsible for processing visual sensory information. It is located posterior to the parieto-occipital sulcus and extends to the preoccipital notch.
A genus of the family CEBIDAE, subfamily CEBINAE, consisting of four species which are divided into two groups, the tufted and untufted. C. apella has tufts of hair over the eyes and sides of the head. The remaining species are without tufts - C. capucinus, C. nigrivultatus, and C. albifrons. Cebus inhabits the forests of Central and South America.
A species of the genus MACACA which typically lives near the coast in tidal creeks and mangrove swamps primarily on the islands of the Malay peninsula.
The relationship between the dose of an administered drug and the response of the organism to the drug.
Studies used to test etiologic hypotheses in which inferences about an exposure to putative causal factors are derived from data relating to characteristics of persons under study or to events or experiences in their past. The essential feature is that some of the persons under study have the disease or outcome of interest and their characteristics are compared with those of unaffected persons.
A method of delineating blood vessels by subtracting a tissue background image from an image of tissue plus intravascular contrast material that attenuates the X-ray photons. The background image is determined from a digitized image taken a few moments before injection of the contrast material. The resulting angiogram is a high-contrast image of the vessel. This subtraction technique allows extraction of a high-intensity signal from the superimposed background information. The image is thus the result of the differential absorption of X-rays by different tissues.
Awareness of oneself in relation to time, place and person.
A species of toxic plants of the Compositae. The poisonous compounds are alkaloids which cause cattle diseases, neoplasms, and liver damage and are used to produce cancers in experimental animals.
The discipline studying genetic composition of populations and effects of factors such as GENETIC SELECTION, population size, MUTATION, migration, and GENETIC DRIFT on the frequencies of various GENOTYPES and PHENOTYPES using a variety of GENETIC TECHNIQUES.
The relative amount by which the average fitness of a POPULATION is lowered, due to the presence of GENES that decrease survival, compared to the GENOTYPE with maximum or optimal fitness. (From Rieger et al., Glossary of Genetics: Classical and Molecular, 5th ed)

The effect of face inversion on activity in human neural systems for face and object perception. (1/1471)

The differential effect of stimulus inversion on face and object recognition suggests that inverted faces are processed by mechanisms for the perception of other objects rather than by face perception mechanisms. We investigated the face inversion using functional magnetic resonance imaging (fMRI). The principal effect of face inversion on was an increased response in ventral extrastriate regions that respond preferentially to another class of objects (houses). In contrast, house inversion did not produce a similar change in face-selective regions. Moreover, stimulus inversion had equivalent, minimal effects for faces in in face-selective regions and for houses in house-selective regions. The results suggest that the failure of face perception systems with inverted faces leads to the recruitment of processing resources in object perception systems, but this failure is not reflected by altered activity in face perception systems.  (+info)

The cerebral haemodynamics of music perception. A transcranial Doppler sonography study. (2/1471)

The perception of music has been investigated by several neurophysiological and neuroimaging methods. Results from these studies suggest a right hemisphere dominance for non-musicians and a possible left hemisphere dominance for musicians. However, inconsistent results have been obtained, and not all variables have been controlled by the different methods. We performed a study with functional transcranial Doppler sonography (fTCD) of the middle cerebral artery to evaluate changes in cerebral blood flow velocity (CBFV) during different periods of music perception. Twenty-four healthy right-handed subjects were enrolled and examined during rest and during listening to periods of music with predominant language, rhythm and harmony content. The gender, musical experience and mode of listening of the subjects were chosen as independent factors; the type of music was included as the variable in repeated measurements. We observed a significant increase of CBFV in the right hemisphere in non-musicians during harmony perception but not during rhythm perception; this effect was more pronounced in females. Language perception was lateralized to the left hemisphere in all subject groups. Musicians showed increased CBFV values in the left hemisphere which were independent of the type of stimulus, and background listeners showed increased CBFV values during harmony perception in the right hemisphere which were independent of their musical experience. The time taken to reach the peak of CBFV was significantly longer in non-musicians when compared with musicians during rhythm and harmony perception. Pulse rates were significantly decreased in non-musicians during harmony perception, probably due to a specific relaxation effect in this subgroup. The resistance index did not show any significant differences, suggesting only regional changes of small resistance vessels but not of large arteries. Our fTCD study confirms previous findings of right hemisphere lateralization for harmony perception in non-musicians. In addition, we showed that this effect is more pronounced in female subjects and in background listeners and that the lateralization is delayed in non-musicians compared with musicians for the perception of rhythm and harmony stimuli. Our data suggest that musicians and non-musicians have different strategies to lateralize musical stimuli, with a delayed but marked right hemisphere lateralization during harmony perception in non-musicians and an attentive mode of listening contributing to a left hemisphere lateralization in musicians.  (+info)

Unilateral neglect and disambiguation of the Necker cube. (3/1471)

Three groups of patients (right brain-damaged patients with or without left neglect, and left brain-damaged patients) and a group of healthy subjects, matched for age and educational level to the three groups of patients, were asked to report which of the two frontal surfaces of Necker cubes oriented in four different ways looked, at first sight, nearer to the viewer. The extent to which, and the way in which, disambiguation of the apparent perspective of Necker cubes occurred was found to vary across the four orientations and to be different in left-neglect patients compared with subjects of the other three groups. With normal subjects, the disambiguating factor is suggested to be a disposition to perceive the upper surface, which is nearly orthogonal to the frontal plane, as external to the cube. This would result from a navigation of the observer's spatial attention towards its target along a particular path that is altered in patients suffering from left neglect. It is suggested that comparison of the paths followed by the attentional vectors of normal subjects and left-neglect patients is potentially fruitful for a better understanding of the brain's normal mechanisms of spatial attention and of unresolved issues concerning the perception of the Necker cube.  (+info)

Contralateral deafness following unilateral suboccipital brain tumor surgery in a patient with large vestibular aqueduct--case report. (4/1471)

A 68-year-old female developed contralateral deafness following extirpation of a left cerebellopontine angle epidermoid cyst. Computed tomography showed that large vestibular aqueduct was present. This unusual complication may have been caused by an abrupt pressure change after cerebrospinal fluid release, which was transmitted through the large vestibular aqueduct and resulted in cochlear damage.  (+info)

The role of retinal waves and synaptic normalization in retinogeniculate development. (5/1471)

The prenatal development of the cat retinogeniculate pathway is thought to involve activity-dependent mechanisms driven by spontaneous waves of retinal activity. The role of these waves upon the segregation of the dorsal lateral geniculate nucleus (LGN) into two eye-specific layers and the development of retinotopic mappings have previously been investigated in a computer model. Using this model, we examine three aspects of retinogeniculate development. First, the mapping of visual space across the whole network into projection columns is shown to be similar to the mapping found in the cat. Second, the simplicity of the model allows us to explore how different forms of synaptic normalization affect development. In comparison to most previous models of ocular dominance, we find that subtractive postsynaptic normalization is redundant and divisive presynaptic normalization is sufficient for normal development. Third, the model predicts that the more often one eye generates waves relative to the other eye, the more LGN units will monocularly respond to the more active eye. In the limit when one eye does not generate any waves, that eye totally disconnects from the LGN allowing the non-deprived eye to innervate all of the LGN. Thus, as well as accounting for normal retinogeniculate development, the model also predicts development under abnormal conditions which can be experimentally tested.  (+info)

Development and organization of ocular dominance bands in primary visual cortex of the sable ferret. (6/1471)

Thalamocortical afferents in the visual cortex of the adult sable ferret are segregated into eye-specific ocular dominance bands. The development of ocular dominance bands was studied by transneuronal labeling of the visual cortices of ferret kits between the ages of postnatal day 28 (P28) and P81 after intravitreous injections of either tritiated proline or wheat germ agglutinin-horseradish peroxidase. Laminar specificity was evident in the youngest animals studied and was similar to that in the adult by P50. In P28 and P30 ferret kits, no modulation reminiscent of ocular dominance bands was detectable in the pattern of labeling along layer IV. By P37 a slight fluctuation in the density of labeling in layer IV was evident in serial reconstructions. By P50, the amplitude of modulation had increased considerably but the pattern of ocular dominance bands did not yet appear mature. The pattern and degree of modulation of the ocular dominance bands resembled that in adult animals by P63. Flat mounts of cortex and serial reconstructions of layer IV revealed an unusual arrangement of inputs serving the two eyes in the region rostral to the periodic ocular dominance bands. In this region, inputs serving the contralateral eye were commonly fused along a mediolateral axis, rostral to which were large and sometimes fused patches of ipsilateral input.  (+info)

Humour appreciation: a role of the right frontal lobe. (7/1471)

Humour occupies a special place in human social interactions. The brain regions and the potential psychological processes underlying humour appreciation were investigated by testing patients who had focal damage in various areas of the brain. A specific brain region, the right frontal lobe, most disrupted the ability to appreciate humour. The individuals with damage in this brain region also reacted less, with diminished physical or emotional responses (laughter, smiling). Performance on the humour appreciation tests used were correlated in a distinct pattern with tests assessing cognitive processes. The ability to hold information in mind (working memory) was related to both verbal (jokes) and non-verbal (cartoon) tests of humour appreciation. In addition, the demands of the specific type of humour test were related in a logical manner to cognitive processes, verbal humour being associated with verbal abstraction ability and mental shifting and cartoon humour being related to the abilities to focus attention to details and to visually search the environment. The ability of the right frontal lobe may be unique in integrating cognitive and affective information, an integration relevant for other complex human abilities, such as episodic memory and self-awareness.  (+info)

Neglect after right insular cortex infarction. (8/1471)

BACKGROUND AND PURPOSE: Case reports have shown an association between right insular damage and neglect. The aim of this study was to examine the incidence of neglect among patient groups with right or left insular infarction. METHODS: We examined neglect in 9 right-handed subjects with insular stroke as evidenced by CT and/or MRI scans (4 with right insular and 5 with left insular cerebrovascular accident) between 4 and 8 weeks after acute stroke with tests of visual, tactile, and auditory perception. RESULTS: Compared with patients with left insular lesions, patients with right insular lesions showed significant neglect in the tactile, auditory, and visual modalities. CONCLUSIONS: The right insular cortex seems to have a role in awareness of external stimuli, and infarction in this area may lead to neglect in multisensory modalities.  (+info)

Cerebral infarction can result in a range of symptoms, including sudden weakness or numbness in the face, arm, or leg on one side of the body, difficulty speaking or understanding speech, sudden vision loss, dizziness, and confusion. Depending on the location and severity of the infarction, it can lead to long-term disability or even death.

There are several types of cerebral infarction, including:

1. Ischemic stroke: This is the most common type of cerebral infarction, accounting for around 87% of all cases. It occurs when a blood clot blocks the flow of blood to the brain, leading to cell death and tissue damage.
2. Hemorrhagic stroke: This type of cerebral infarction occurs when a blood vessel in the brain ruptures, leading to bleeding and cell death.
3. Lacunar infarction: This type of cerebral infarction affects the deep structures of the brain, particularly the basal ganglia, and is often caused by small blockages or stenosis (narrowing) in the blood vessels.
4. Territorial infarction: This type of cerebral infarction occurs when there is a complete blockage of a blood vessel that supplies a specific area of the brain, leading to cell death and tissue damage in that area.

Diagnosis of cerebral infarction typically involves a combination of physical examination, medical history, and imaging tests such as CT or MRI scans. Treatment options vary depending on the cause and location of the infarction, but may include medication to dissolve blood clots, surgery to remove blockages, or supportive care to manage symptoms and prevent complications.

Causes:

1. Brain injury during fetal development or birth
2. Hypoxia (oxygen deficiency) to the brain, often due to complications during labor and delivery
3. Infections such as meningitis or encephalitis
4. Stroke or bleeding in the brain
5. Traumatic head injury
6. Genetic disorders
7. Premature birth
8. Low birth weight
9. Multiples (twins, triplets)
10. Maternal infections during pregnancy.

Symptoms:

1. Weakness or paralysis of muscles on one side of the body
2. Lack of coordination and balance
3. Difficulty with movement, posture, and gait
4. Spasticity (stiffness) or hypotonia (looseness) of muscles
5. Intellectual disability or learning disabilities
6. Seizures
7. Vision, hearing, or speech problems
8. Swallowing difficulties
9. Increased risk of infections and bone fractures
10. Delays in reaching developmental milestones.

Diagnosis:

1. Physical examination and medical history
2. Imaging tests, such as CT or MRI scans
3. Electromyography (EMG) to test muscle activity
4. Developmental assessments to evaluate cognitive and motor skills
5. Genetic testing to identify underlying causes.

Treatment:

1. Physical therapy to improve movement, balance, and strength
2. Occupational therapy to develop daily living skills and fine motor activities
3. Speech therapy for communication and swallowing difficulties
4. Medications to control seizures, spasticity, or pain
5. Surgery to correct anatomical abnormalities or release contracted muscles
6. Assistive devices, such as braces, walkers, or wheelchairs, to aid mobility and independence.

It's important to note that each individual with Cerebral Palsy may have a unique combination of symptoms and require a personalized treatment plan. With appropriate medical care and support, many individuals with Cerebral Palsy can lead fulfilling lives and achieve their goals despite the challenges they face.

Infarction Middle Cerebral Artery (MCA) is a type of ischemic stroke that occurs when there is an obstruction in the middle cerebral artery. This artery supplies blood to the temporal lobe of the brain, which controls many important functions such as memory, language, and spatial reasoning. When this artery becomes blocked or ruptured, it can cause a lack of blood supply to the affected areas resulting in tissue death (infarction).

The symptoms of an MCA infarction can vary depending on the location and severity of the blockage. Some common symptoms include weakness or paralysis on one side of the body, difficulty with speech and language, memory loss, confusion, vision problems, and difficulty with coordination and balance. Patients may also experience sudden severe headache, nausea, vomiting, and fever.

The diagnosis of MCA infarction is based on a combination of clinical examination, imaging studies such as CT or MRI scans, and laboratory tests. Imaging studies can help to identify the location and severity of the blockage, while laboratory tests may be used to rule out other conditions that may cause similar symptoms.

Treatment for MCA infarction depends on the underlying cause of the blockage or rupture. In some cases, medications such as thrombolytics may be given to dissolve blood clots and restore blood flow to the affected areas. Surgery may also be required to remove any blockages or repair damaged blood vessels. Other interventions such as endovascular procedures or brain bypass surgery may also be used to restore blood flow.

In summary, middle cerebral artery infarction is a type of stroke that occurs when the blood supply to the brain is blocked or interrupted, leading to damage to the brain tissue. It can cause a range of symptoms including weakness or paralysis on one side of the body, difficulty with speech and language, memory loss, confusion, vision problems, and difficulty with coordination and balance. The diagnosis is based on a combination of clinical examination, imaging studies, and laboratory tests. Treatment options include medications, surgery, endovascular procedures, or brain bypass surgery.

Cerebral malaria occurs when the parasites that cause malaria (Plasmodium falciparum) infect and multiply in red blood cells in the brain, causing inflammation and damage to brain tissue. This can lead to a range of symptoms including seizures, coma, and even death.

Cerebral malaria is diagnosed through a combination of physical examination, laboratory tests (such as blood smears or PCR), and imaging studies (such as CT or MRI scans). Treatment typically involves the use of antimalarial drugs, such as artemisinin-based combination therapies (ACTs) or quinine, which can help to clear the parasites from the bloodstream and reduce inflammation in the brain. In severe cases, treatment may also involve supportive care, such as mechanical ventilation or dialysis, to manage complications related to the disease.

Prevention of cerebral malaria is challenging, but measures such as using insecticide-treated bed nets, wearing protective clothing, and applying insect repellents can help reduce the risk of infection. Eliminating standing water around homes and communities where mosquitoes can breed can also help reduce the incidence of malaria.

The prognosis for cerebral malaria varies depending on the severity of the disease and the promptness and effectiveness of treatment. In general, early diagnosis and treatment improve outcomes, while delayed or inadequate treatment can lead to serious complications or death. According to the World Health Organization (WHO), cerebral malaria is a leading cause of mortality in African children under the age of five, with an estimated 20% to 30% mortality rate in severe cases.

The term ischemia refers to the reduction of blood flow, and it is often used interchangeably with the term stroke. However, not all strokes are caused by ischemia, as some can be caused by other factors such as bleeding in the brain. Ischemic stroke accounts for about 87% of all strokes.

There are different types of brain ischemia, including:

1. Cerebral ischemia: This refers to the reduction of blood flow to the cerebrum, which is the largest part of the brain and responsible for higher cognitive functions such as thought, emotion, and voluntary movement.
2. Cerebellar ischemia: This refers to the reduction of blood flow to the cerebellum, which is responsible for coordinating and regulating movement, balance, and posture.
3. Brainstem ischemia: This refers to the reduction of blood flow to the brainstem, which is responsible for controlling many of the body's automatic functions such as breathing, heart rate, and blood pressure.
4. Territorial ischemia: This refers to the reduction of blood flow to a specific area of the brain, often caused by a blockage in a blood vessel.
5. Global ischemia: This refers to the reduction of blood flow to the entire brain, which can be caused by a cardiac arrest or other systemic conditions.

The symptoms of brain ischemia can vary depending on the location and severity of the condition, but may include:

1. Weakness or paralysis of the face, arm, or leg on one side of the body
2. Difficulty speaking or understanding speech
3. Sudden vision loss or double vision
4. Dizziness or loss of balance
5. Confusion or difficulty with memory
6. Seizures
7. Slurred speech or inability to speak
8. Numbness or tingling sensations in the face, arm, or leg
9. Vision changes, such as blurred vision or loss of peripheral vision
10. Difficulty with coordination and balance.

It is important to seek medical attention immediately if you experience any of these symptoms, as brain ischemia can cause permanent damage or death if left untreated.

Symptoms of cerebral hemorrhage may include sudden severe headache, confusion, seizures, weakness or numbness in the face or limbs, and loss of consciousness. The condition is diagnosed through a combination of physical examination, imaging tests such as CT or MRI scans, and laboratory tests to determine the cause of the bleeding.

Treatment for cerebral hemorrhage depends on the location and severity of the bleeding, as well as the underlying cause. Medications may be used to control symptoms such as high blood pressure or seizures, while surgery may be necessary to repair the ruptured blood vessel or relieve pressure on the brain. In some cases, the condition may be fatal, and immediate medical attention is essential to prevent long-term damage or death.

Some of the most common complications associated with cerebral hemorrhage include:

1. Rebleeding: There is a risk of rebleeding after the initial hemorrhage, which can lead to further brain damage and increased risk of death.
2. Hydrocephalus: Excess cerebrospinal fluid can accumulate in the brain, leading to increased intracranial pressure and potentially life-threatening complications.
3. Brain edema: Swelling of the brain tissue can occur due to the bleeding, leading to increased intracranial pressure and potentially life-threatening complications.
4. Seizures: Cerebral hemorrhage can cause seizures, which can be a sign of a more severe injury.
5. Cognitive and motor deficits: Depending on the location and severity of the bleeding, cerebral hemorrhage can result in long-term cognitive and motor deficits.
6. Vision loss: Cerebral hemorrhage can cause vision loss or blindness due to damage to the visual cortex.
7. Communication difficulties: Cerebral hemorrhage can cause difficulty with speech and language processing, leading to communication difficulties.
8. Behavioral changes: Depending on the location and severity of the bleeding, cerebral hemorrhage can result in behavioral changes, such as irritability, agitation, or apathy.
9. Infection: Cerebral hemorrhage can increase the risk of infection, particularly if the hemorrhage is caused by a ruptured aneurysm or arteriovenous malformation (AVM).
10. Death: Cerebral hemorrhage can be fatal, particularly if the bleeding is severe or if there are underlying medical conditions that compromise the patient's ability to tolerate the injury.

Example sentence: "The patient experienced a transient ischemic attack, which was caused by a temporary blockage in one of the blood vessels in their brain."

Synonyms: TIA, mini-stroke.

The term "cerebral" refers to the brain, "amyloid" refers to the abnormal protein deposits, and "angiopathy" refers to the damage caused to the blood vessels. CAA is often associated with other conditions such as Alzheimer's disease, Down syndrome, and other forms of dementia.

CAA is a type of small vessel ischemic disease (SVID), which affects the smaller blood vessels in the brain. The exact cause of CAA is not yet fully understood, but it is thought to be related to a combination of genetic and environmental factors. There is currently no cure for CAA, but researchers are working to develop new treatments to slow its progression and manage its symptoms.

Some common symptoms of CAA include:

* Cognitive decline
* Seizures
* Stroke-like episodes
* Memory loss
* Confusion
* Difficulty with coordination and balance

If you suspect you or a loved one may be experiencing symptoms of CAA, it is important to speak with a healthcare professional for proper diagnosis and treatment. A thorough medical history and physical examination, along with imaging tests such as MRI or CT scans, can help confirm the presence of CAA.

While there is no cure for CAA, there are several treatment options available to manage its symptoms and slow its progression. These may include medications to control seizures, improve cognitive function, and reduce inflammation. In some cases, surgery or endovascular procedures may be necessary to repair or remove damaged blood vessels.

It is important to note that CAA is a complex condition, and its management requires a multidisciplinary approach involving neurologists, geriatricians, radiologists, and other healthcare professionals. With proper diagnosis and treatment, however, many individuals with CAA are able to lead active and fulfilling lives.

The word "edema" comes from the Greek word "oidema", meaning swelling.

1. Stroke: A stroke occurs when the blood supply to the brain is interrupted, either due to a blockage or a rupture of the blood vessels. This can lead to cell death and permanent brain damage.
2. Cerebral vasospasm: Vasospasm is a temporary constriction of the blood vessels in the brain, which can occur after a subarachnoid hemorrhage (bleeding in the space surrounding the brain).
3. Moyamoya disease: This is a rare condition caused by narrowing or blockage of the internal carotid artery and its branches. It can lead to recurrent transient ischemic attacks (TIs) or stroke.
4. Cerebral amyloid angiopathy: This is a condition where abnormal protein deposits accumulate in the blood vessels of the brain, leading to inflammation and bleeding.
5. Cavernous malformations: These are abnormal collections of blood vessels in the brain that can cause seizures, headaches, and other symptoms.
6. Carotid artery disease: Atherosclerosis (hardening) of the carotid arteries can lead to a stroke or TIAs.
7. Vertebrobasilar insufficiency: This is a condition where the blood flow to the brain is reduced due to narrowing or blockage of the vertebral and basilar arteries.
8. Temporal lobe dementia: This is a type of dementia that affects the temporal lobe of the brain, leading to memory loss and other cognitive symptoms.
9. Cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy (CADASIL): This is a rare genetic disorder that affects the blood vessels in the brain, leading to recurrent stroke-like events.
10. Moyamoya disease: This is a rare condition caused by narrowing or blockage of the internal carotid artery and its branches, leading to decreased blood flow to the brain and increased risk of stroke.

It's important to note that this list is not exhaustive and there may be other causes of stroke and TIAs that are not included here. A proper diagnosis can only be made by a qualified medical professional after conducting a thorough examination and reviewing the individual's medical history.

1. Atrial fibrillation (a type of irregular heartbeat)
2. Heart disease or valve problems
3. Blood clots in the legs or lungs
4. Infective endocarditis (an infection of the heart valves)
5. Cancer and its treatment
6. Trauma to the head or neck
7. High blood pressure
8. Atherosclerosis (the buildup of plaque in the arteries)

When a blockage occurs in one of the blood vessels of the brain, it can deprive the brain of oxygen and nutrients, leading to cell death and potentially causing a range of symptoms including:

1. Sudden weakness or numbness in the face, arm, or leg
2. Sudden confusion or trouble speaking or understanding speech
3. Sudden trouble seeing in one or both eyes
4. Sudden severe headache
5. Dizziness or loss of balance
6. Fainting or falling

Intracranial embolism and thrombosis can be diagnosed through a variety of imaging tests, including:

1. Computed tomography (CT) scan
2. Magnetic resonance imaging (MRI)
3. Magnetic resonance angiography (MRA)
4. Cerebral angiography
5. Doppler ultrasound

Treatment options for intracranial embolism and thrombosis depend on the underlying cause of the blockage, but may include:

1. Medications to dissolve blood clots or prevent further clotting
2. Surgery to remove the blockage or repair the affected blood vessel
3. Endovascular procedures, such as angioplasty and stenting, to open up narrowed or blocked blood vessels
4. Supportive care, such as oxygen therapy and pain management, to help manage symptoms and prevent complications.

Some common types of brain diseases include:

1. Neurodegenerative diseases: These are progressive conditions that damage or kill brain cells over time, leading to memory loss, cognitive decline, and movement disorders. Examples include Alzheimer's disease, Parkinson's disease, Huntington's disease, and amyotrophic lateral sclerosis (ALS).
2. Stroke: This occurs when blood flow to the brain is interrupted, leading to cell death and potential long-term disability.
3. Traumatic brain injury (TBI): This refers to any type of head injury that causes damage to the brain, such as concussions, contusions, or penetrating wounds.
4. Infections: Viral, bacterial, and fungal infections can all affect the brain, leading to a range of symptoms including fever, seizures, and meningitis.
5. Tumors: Brain tumors can be benign or malignant and can cause a variety of symptoms depending on their location and size.
6. Cerebrovascular diseases: These conditions affect the blood vessels of the brain, leading to conditions such as aneurysms, arteriovenous malformations (AVMs), and Moyamoya disease.
7. Neurodevelopmental disorders: These are conditions that affect the development of the brain and nervous system, such as autism spectrum disorder, ADHD, and intellectual disability.
8. Sleep disorders: Conditions such as insomnia, narcolepsy, and sleep apnea can all have a significant impact on brain function.
9. Psychiatric disorders: Mental health conditions such as depression, anxiety, and schizophrenia can affect the brain and its functioning.
10. Neurodegenerative with brain iron accumulation: Conditions such as Parkinson's disease, Alzheimer's disease, and Huntington's disease are characterized by the accumulation of abnormal proteins and other substances in the brain, leading to progressive loss of brain function over time.

It is important to note that this is not an exhaustive list and there may be other conditions or factors that can affect the brain and its functioning. Additionally, many of these conditions can have a significant impact on a person's quality of life, and it is important to seek medical attention if symptoms persist or worsen over time.

Intracranial aneurysms are relatively rare but can have serious consequences if they rupture and cause bleeding in the brain.

The symptoms of an unruptured intracranial aneurysm may include headaches, seizures, and visual disturbances.

If an intracranial aneurysm ruptures, it can lead to a subarachnoid hemorrhage (bleeding in the space around the brain), which is a medical emergency that requires immediate treatment.

Diagnosis of an intracranial aneurysm typically involves imaging tests such as CT or MRI scans, and may also involve catheter angiography.

Treatment for intracranial aneurysms usually involves surgical clipping or endovascular coiling, depending on the size, location, and severity of the aneurysm.

Preventing rupture of intracranial aneurysms is important, as they can be difficult to treat once they have ruptured.

Endovascular coiling is a minimally invasive procedure in which a catheter is inserted into the affected artery and a small coil is inserted into the aneurysm, causing it to clot and preventing further bleeding.

Surgical clipping involves placing a small metal clip across the base of the aneurysm to prevent further bleeding.

In addition to these treatments, medications such as anticonvulsants and antihypertensives may be used to manage symptoms and prevent complications.

If you suspect vasospasm, it is essential to seek medical attention immediately. A healthcare professional will perform a physical examination and order imaging tests, such as CT or MRI scans, to confirm the diagnosis. Treatment options may include medications to dilate blood vessels, surgery to relieve pressure on affected areas, or other interventions depending on the severity of the condition.

Preventing vasospasm can be challenging, but some measures can reduce the risk of developing this condition. These include managing underlying conditions such as high blood pressure, diabetes, or high cholesterol levels; avoiding head injuries by wearing protective gear during sports and other activities; and adopting a healthy lifestyle that includes regular exercise and a balanced diet.

Early diagnosis and treatment are critical in managing vasospasm and preventing long-term damage to the brain tissue. If you experience any symptoms suggestive of vasospasm, seek medical attention promptly to receive appropriate care and improve outcomes.

Brain hypoxia is a serious medical condition that requires prompt treatment to prevent long-term damage and improve outcomes for patients. Treatment options may include oxygen therapy, medications to improve blood flow to the brain, and surgery to remove any blockages or obstructions in blood vessels.

1) They share similarities with humans: Many animal species share similar biological and physiological characteristics with humans, making them useful for studying human diseases. For example, mice and rats are often used to study diseases such as diabetes, heart disease, and cancer because they have similar metabolic and cardiovascular systems to humans.

2) They can be genetically manipulated: Animal disease models can be genetically engineered to develop specific diseases or to model human genetic disorders. This allows researchers to study the progression of the disease and test potential treatments in a controlled environment.

3) They can be used to test drugs and therapies: Before new drugs or therapies are tested in humans, they are often first tested in animal models of disease. This allows researchers to assess the safety and efficacy of the treatment before moving on to human clinical trials.

4) They can provide insights into disease mechanisms: Studying disease models in animals can provide valuable insights into the underlying mechanisms of a particular disease. This information can then be used to develop new treatments or improve existing ones.

5) Reduces the need for human testing: Using animal disease models reduces the need for human testing, which can be time-consuming, expensive, and ethically challenging. However, it is important to note that animal models are not perfect substitutes for human subjects, and results obtained from animal studies may not always translate to humans.

6) They can be used to study infectious diseases: Animal disease models can be used to study infectious diseases such as HIV, TB, and malaria. These models allow researchers to understand how the disease is transmitted, how it progresses, and how it responds to treatment.

7) They can be used to study complex diseases: Animal disease models can be used to study complex diseases such as cancer, diabetes, and heart disease. These models allow researchers to understand the underlying mechanisms of the disease and test potential treatments.

8) They are cost-effective: Animal disease models are often less expensive than human clinical trials, making them a cost-effective way to conduct research.

9) They can be used to study drug delivery: Animal disease models can be used to study drug delivery and pharmacokinetics, which is important for developing new drugs and drug delivery systems.

10) They can be used to study aging: Animal disease models can be used to study the aging process and age-related diseases such as Alzheimer's and Parkinson's. This allows researchers to understand how aging contributes to disease and develop potential treatments.

1. Ischemic stroke: This is the most common type of stroke, accounting for about 87% of all strokes. It occurs when a blood vessel in the brain becomes blocked, reducing blood flow to the brain.
2. Hemorrhagic stroke: This type of stroke occurs when a blood vessel in the brain ruptures, causing bleeding in the brain. High blood pressure, aneurysms, and blood vessel malformations can all cause hemorrhagic strokes.
3. Transient ischemic attack (TIA): Also known as a "mini-stroke," a TIA is a temporary interruption of blood flow to the brain that lasts for a short period of time, usually less than 24 hours. TIAs are often a warning sign for a future stroke and should be taken seriously.

Stroke can cause a wide range of symptoms depending on the location and severity of the damage to the brain. Some common symptoms include:

* Weakness or numbness in the face, arm, or leg
* Difficulty speaking or understanding speech
* Sudden vision loss or double vision
* Dizziness, loss of balance, or sudden falls
* Severe headache
* Confusion, disorientation, or difficulty with memory

Stroke is a leading cause of long-term disability and can have a significant impact on the quality of life for survivors. However, with prompt medical treatment and rehabilitation, many people are able to recover some or all of their lost functions and lead active lives.

The medical community has made significant progress in understanding stroke and developing effective treatments. Some of the most important advances include:

* Development of clot-busting drugs and mechanical thrombectomy devices to treat ischemic strokes
* Improved imaging techniques, such as CT and MRI scans, to diagnose stroke and determine its cause
* Advances in surgical techniques for hemorrhagic stroke
* Development of new medications to prevent blood clots and reduce the risk of stroke

Despite these advances, stroke remains a significant public health problem. According to the American Heart Association, stroke is the fifth leading cause of death in the United States and the leading cause of long-term disability. In 2017, there were over 795,000 strokes in the United States alone.

There are several risk factors for stroke that can be controlled or modified. These include:

* High blood pressure
* Diabetes mellitus
* High cholesterol levels
* Smoking
* Obesity
* Lack of physical activity
* Poor diet

In addition to these modifiable risk factors, there are also several non-modifiable risk factors for stroke, such as age (stroke risk increases with age), family history of stroke, and previous stroke or transient ischemic attack (TIA).

The medical community has made significant progress in understanding the causes and risk factors for stroke, as well as developing effective treatments and prevention strategies. However, more research is needed to improve outcomes for stroke survivors and reduce the overall burden of this disease.

There are several types of intracranial thrombosis, including:

1. Cerebral venous sinus thrombosis (CVST): This type of thrombosis occurs when a blood clot forms in the veins that drain blood from the brain. CVST is more common in young adults and is often associated with certain risk factors, such as cancer, infection, or trauma.
2. Cerebral arterial thrombosis (CAT): This type of thrombosis occurs when a blood clot forms in an artery that supplies blood to the brain. CAT is more common in older adults and is often associated with risk factors such as high blood pressure, diabetes, or heart disease.
3. Pial sinus thrombosis: This type of thrombosis occurs when a blood clot forms in the pial sinuses, which are specialized blood vessels that surround the brain. Pial sinus thrombosis is more common in children and young adults.

The symptoms of intracranial thrombosis can vary depending on the location and size of the clot, but may include:

1. Headache: A severe headache is often the first symptom of intracranial thrombosis. The headache may be sudden and severe, or it may develop gradually over time.
2. Confusion: Patients with intracranial thrombosis may experience confusion, disorientation, or difficulty concentrating.
3. Weakness or numbness: Patients may experience weakness or numbness in their arms, legs, or face on one side of the body.
4. Vision problems: Intracranial thrombosis can cause vision problems, such as blurred vision, double vision, or loss of peripheral vision.
5. Speech difficulties: Patients may experience difficulty speaking or understanding speech.
6. Seizures: In some cases, intracranial thrombosis can cause seizures.
7. Fever: Patients with intracranial thrombosis may develop a fever, especially if the clot is infected.
8. Weakness in the limbs: Patients may experience weakness or paralysis in their arms or legs.
9. Difficulty swallowing: Patients may have difficulty swallowing or experience drooling.
10. Change in mental status: Patients with intracranial thrombosis may exhibit changes in their mental status, such as lethargy, agitation, or confusion.

If you or someone you know is experiencing these symptoms, it is important to seek medical attention immediately. Intracranial thrombosis can be diagnosed through imaging tests such as CT or MRI scans, and treated with anticoagulant medications, thrombolysis, or surgery. Early diagnosis and treatment can help prevent long-term damage and improve outcomes for patients.

Types: There are several types of brain infarction, including:

1. Cerebral infarction: This type of infarction occurs when there is a blockage or obstruction in the blood vessels that supply the cerebrum, which is the largest part of the brain.
2. Cerebellar infarction: This type of infarction occurs when there is a blockage or obstruction in the blood vessels that supply the cerebellum, which is located at the base of the brain.
3. Brain stem infarction: This type of infarction occurs when there is a blockage or obstruction in the blood vessels that supply the brain stem, which is the part of the brain that controls vital functions such as breathing, heart rate, and blood pressure.

Symptoms: The symptoms of brain infarction can vary depending on the location and size of the affected area, but common symptoms include:

1. Sudden weakness or numbness in the face, arm, or leg
2. Sudden confusion or trouble speaking or understanding speech
3. Sudden difficulty seeing or blindness
4. Sudden difficulty walking or loss of balance
5. Sudden severe headache
6. Difficulty with coordination and movement
7. Slurred speech
8. Vision changes
9. Seizures

Diagnosis: Brain infarction is typically diagnosed using a combination of physical examination, medical history, and imaging tests such as CT or MRI scans. Other diagnostic tests may include blood tests to check for signs of infection or blood clotting abnormalities, and an electroencephalogram (EEG) to measure the electrical activity of the brain.

Treatment: The treatment of brain infarction depends on the underlying cause, but common treatments include:

1. Medications: To control symptoms such as high blood pressure, seizures, and swelling in the brain.
2. Endovascular therapy: A minimally invasive procedure to open or remove blockages in the blood vessels.
3. Surgery: To relieve pressure on the brain or repair damaged blood vessels.
4. Rehabilitation: To help regain lost function and improve quality of life.

Prognosis: The prognosis for brain infarction depends on the location and size of the affected area, as well as the promptness and effectiveness of treatment. In general, the earlier treatment is received, the better the outcome. However, some individuals may experience long-term or permanent disability, or even death.

There are several types of intracranial AVMs, including:

1. Cerebral AVMs: These are the most common type of AVM and occur in the cerebral hemispheres of the brain.
2. Spinal AVMs: These occur in the spinal cord and are less common than cerebral AVMs.
3. Multiple AVMs: Some people may have multiple AVMs, which can be located in different parts of the brain or spine.

The symptoms of intracranial AVMs can vary depending on the location and size of the malformation. They may include:

1. Seizures: AVMs can cause seizures, which can be a sign of the malformation.
2. Headaches: Patients with AVMs may experience frequent and severe headaches.
3. Weakness or numbness: AVMs can cause weakness or numbness in the arms or legs.
4. Vision problems: AVMs can affect the vision, including blurriness, double vision, or loss of peripheral vision.
5. Confusion or disorientation: Patients with AVMs may experience confusion or disorientation.
6. Seizures: AVMs can cause seizures, which can be a sign of the malformation.
7. Cranial nerve deficits: AVMs can affect the cranial nerves, leading to problems with speech, hearing, or facial movements.
8. Hydrocephalus: AVMs can cause hydrocephalus, which is an accumulation of fluid in the brain.

The diagnosis of intracranial AVMs is based on a combination of clinical symptoms, neuroimaging studies such as CT or MRI scans, and angiography. Angiography is a test that uses dye and X-rays to visualize the blood vessels in the brain.

Treatment of intracranial AVMs usually involves a multidisciplinary approach, including neurosurgeons, interventional neuroradiologists, and neurologists. Treatment options may include:

1. Observation: Small AVMs that are not causing symptoms may be monitored with regular imaging studies to see if they grow or change over time.
2. Endovascular embolization: This is a minimally invasive procedure in which a catheter is inserted through a blood vessel in the leg and directed to the AVM in the brain. Once there, the catheter releases tiny particles that block the flow of blood into the AVM, causing it to shrink or disappear.
3. Surgery: In some cases, surgery may be necessary to remove the AVM. This is usually done when the AVM is large or in a location that makes it difficult to treat with endovascular embolization.
4. Radiation therapy: This may be used to shrink the AVM before surgery or as a standalone treatment.
5. Chemotherapy: This may be used in combination with radiation therapy to treat AVMs that are caused by a genetic condition called hereditary hemorrhagic telangiectasia (HHT).

The choice of treatment depends on the location and size of the AVM, as well as the patient's overall health and other medical conditions. In some cases, a combination of treatments may be necessary to achieve the best outcome.

Types of Arterial Occlusive Diseases:

1. Atherosclerosis: Atherosclerosis is a condition where plaque builds up inside the arteries, leading to narrowing or blockages that can restrict blood flow to certain areas of the body.
2. Peripheral Artery Disease (PAD): PAD is a condition where the blood vessels in the legs and arms become narrowed or blocked, leading to pain or cramping in the affected limbs.
3. Coronary Artery Disease (CAD): CAD is a condition where the coronary arteries, which supply blood to the heart, become narrowed or blocked, leading to chest pain or a heart attack.
4. Carotid Artery Disease: Carotid artery disease is a condition where the carotid arteries, which supply blood to the brain, become narrowed or blocked, leading to stroke or mini-stroke.
5. Renal Artery Stenosis: Renal artery stenosis is a condition where the blood vessels that supply the kidneys become narrowed or blocked, leading to high blood pressure and decreased kidney function.

Symptoms of Arterial Occlusive Diseases:

1. Pain or cramping in the affected limbs
2. Weakness or fatigue
3. Difficulty walking or standing
4. Chest pain or discomfort
5. Shortness of breath
6. Dizziness or lightheadedness
7. Stroke or mini-stroke

Treatment for Arterial Occlusive Diseases:

1. Medications: Medications such as blood thinners, cholesterol-lowering drugs, and blood pressure medications may be prescribed to treat arterial occlusive diseases.
2. Lifestyle Changes: Lifestyle changes such as quitting smoking, exercising regularly, and eating a healthy diet can help manage symptoms and slow the progression of the disease.
3. Endovascular Procedures: Endovascular procedures such as angioplasty and stenting may be performed to open up narrowed or blocked blood vessels.
4. Surgery: In some cases, surgery may be necessary to treat arterial occlusive diseases, such as bypass surgery or carotid endarterectomy.

Prevention of Arterial Occlusive Diseases:

1. Maintain a healthy diet and lifestyle
2. Quit smoking and avoid exposure to secondhand smoke
3. Exercise regularly
4. Manage high blood pressure, high cholesterol, and diabetes
5. Avoid excessive alcohol consumption
6. Get regular check-ups with your healthcare provider

Early detection and treatment of arterial occlusive diseases can help manage symptoms, slow the progression of the disease, and prevent complications such as heart attack or stroke.

The parasite enters the body through the ingestion of contaminated food or water, and can cause a wide range of symptoms in people with healthy immune systems, including fever, headache, and swollen lymph nodes. However, those with compromised immune systems are more susceptible to severe symptoms, including seizures, confusion, and coma.

Diagnosis of cerebral toxoplasmosis is often made through a combination of physical examination, laboratory tests (such as PCR or IgG antibody detection), and imaging studies (such as CT or MRI scans). Treatment typically involves a combination of antiparasitic medications and supportive care to manage symptoms and prevent complications.

In severe cases, cerebral toxoplasmosis can lead to long-term neurological damage, including cognitive impairment and seizure disorders. Prevention of the disease is primarily focused on avoiding exposure to the parasite, which can be achieved through good hygiene practices (such as proper handling and cooking of meat) and avoiding contact with cat feces, which are a common source of infection.

Overall, cerebral toxoplasmosis is a serious opportunistic infection that can have significant neurological consequences in individuals with compromised immune systems. Prompt diagnosis and appropriate treatment are essential for preventing long-term complications and improving outcomes.

There are several different types of brain injuries that can occur, including:

1. Concussions: A concussion is a type of mild traumatic brain injury that occurs when the brain is jolted or shaken, often due to a blow to the head.
2. Contusions: A contusion is a bruise on the brain that can occur when the brain is struck by an object, such as during a car accident.
3. Coup-contrecoup injuries: This type of injury occurs when the brain is injured as a result of the force of the body striking another object, such as during a fall.
4. Penetrating injuries: A penetrating injury occurs when an object pierces the brain, such as during a gunshot wound or stab injury.
5. Blast injuries: This type of injury occurs when the brain is exposed to a sudden and explosive force, such as during a bombing.

The symptoms of brain injuries can vary depending on the severity of the injury and the location of the damage in the brain. Some common symptoms include:

* Headaches
* Dizziness or loss of balance
* Confusion or disorientation
* Memory loss or difficulty with concentration
* Slurred speech or difficulty with communication
* Vision problems, such as blurred vision or double vision
* Sleep disturbances
* Mood changes, such as irritability or depression
* Personality changes
* Difficulty with coordination and balance

In some cases, brain injuries can be treated with medication, physical therapy, and other forms of rehabilitation. However, in more severe cases, the damage may be permanent and long-lasting. It is important to seek medical attention immediately if symptoms persist or worsen over time.

Anisometropia is typically diagnosed with a comprehensive eye exam, which includes visual acuity testing, refraction, and retinoscopy. Treatment options for anisometropia depend on the underlying cause and severity of the condition, and may include glasses or contact lenses, prism lenses, or surgery. In some cases, anisometropia can be treated with orthokeratology (OK) or corneal reshaping, which involves wearing a specialized contact lens at night to reshape the cornea and improve vision during the day.

Anisometropia is relatively rare, but it can have a significant impact on quality of life, particularly in children and young adults. If you suspect that you or your child may have anisometropia, it's important to schedule an eye exam as soon as possible to determine the underlying cause and develop an appropriate treatment plan.

Hypercapnia is a medical condition where there is an excessive amount of carbon dioxide (CO2) in the bloodstream. This can occur due to various reasons such as:

1. Respiratory failure: When the lungs are unable to remove enough CO2 from the body, leading to an accumulation of CO2 in the bloodstream.
2. Lung disease: Certain lung diseases such as chronic obstructive pulmonary disease (COPD) or pneumonia can cause hypercapnia by reducing the ability of the lungs to exchange gases.
3. Medication use: Certain medications, such as anesthetics and sedatives, can slow down breathing and lead to hypercapnia.

The symptoms of hypercapnia can vary depending on the severity of the condition, but may include:

1. Headaches
2. Dizziness
3. Confusion
4. Shortness of breath
5. Fatigue
6. Sleep disturbances

If left untreated, hypercapnia can lead to more severe complications such as:

1. Respiratory acidosis: When the body produces too much acid, leading to a drop in blood pH.
2. Cardiac arrhythmias: Abnormal heart rhythms can occur due to the increased CO2 levels in the bloodstream.
3. Seizures: In severe cases of hypercapnia, seizures can occur due to the changes in brain chemistry caused by the excessive CO2.

Treatment for hypercapnia typically involves addressing the underlying cause and managing symptoms through respiratory support and other therapies as needed. This may include:

1. Oxygen therapy: Administering oxygen through a mask or nasal tubes to help increase oxygen levels in the bloodstream and reduce CO2 levels.
2. Ventilation assistance: Using a machine to assist with breathing, such as a ventilator, to help remove excess CO2 from the lungs.
3. Carbon dioxide removal: Using a device to remove CO2 from the bloodstream, such as a dialysis machine.
4. Medication management: Adjusting medications that may be contributing to hypercapnia, such as anesthetics or sedatives.
5. Respiratory therapy: Providing breathing exercises and other techniques to help improve lung function and reduce symptoms.

It is important to seek medical attention if you suspect you or someone else may have hypercapnia, as early diagnosis and treatment can help prevent complications and improve outcomes.

Reperfusion injury can cause inflammation, cell death, and impaired function in the affected tissue or organ. The severity of reperfusion injury can vary depending on the duration and severity of the initial ischemic event, as well as the promptness and effectiveness of treatment to restore blood flow.

Reperfusion injury can be a complicating factor in various medical conditions, including:

1. Myocardial infarction (heart attack): Reperfusion injury can occur when blood flow is restored to the heart muscle after a heart attack, leading to inflammation and cell death.
2. Stroke: Reperfusion injury can occur when blood flow is restored to the brain after an ischemic stroke, leading to inflammation and damage to brain tissue.
3. Organ transplantation: Reperfusion injury can occur when a transplanted organ is subjected to ischemia during harvesting or preservation, and then reperfused with blood.
4. Peripheral arterial disease: Reperfusion injury can occur when blood flow is restored to a previously occluded peripheral artery, leading to inflammation and damage to the affected tissue.

Treatment of reperfusion injury often involves medications to reduce inflammation and oxidative stress, as well as supportive care to manage symptoms and prevent further complications. In some cases, experimental therapies such as stem cell transplantation or gene therapy may be used to promote tissue repair and regeneration.

Here are some examples of how 'Aneurysm, Ruptured' is used in different contexts:

1. Medical literature: "The patient was rushed to the hospital with a ruptured aneurysm after experiencing sudden severe headaches and vomiting."
2. Doctor-patient communication: "You have a ruptured aneurysm, which means that your blood vessel has burst and is causing bleeding inside your body."
3. Medical research: "The study found that patients with a history of smoking are at increased risk of developing a ruptured aneurysm."
4. Emergency medical services: "The patient was transported to the hospital with a ruptured aneurysm and was in critical condition upon arrival."
5. Patient education: "To prevent a ruptured aneurysm, it is important to manage high blood pressure and avoid smoking."

Here are some possible clinical presentations and diagnostic procedures for intracranial sinus thrombosis:

Clinical Presentations:

* Headache (most common symptom)
* Fever
* Nasal congestion or swelling
* Pain in the face, particularly on one side
* Vision changes or blurriness
* Nausea and vomiting

Diagnostic Procedures:

1. Imaging studies (CT or MRI scans) to confirm the presence of a blood clot within a sinus and to rule out other conditions that may cause similar symptoms.
2. Endoscopy, which involves inserting a flexible tube with a camera into the nasal cavity to visualize the inside of the sinuses and to collect tissue or fluid samples for further examination.
3. Blood tests to check for infection or inflammation.
4. Sinus aspiration, which involves draining fluid from the affected sinus to determine if there is a blood clot present.

Treatment options for intracranial sinus thrombosis depend on the severity of the condition and may include antibiotics, anticoagulation medications, or surgical drainage of the affected sinus. In some cases, the condition may be life-threatening and require emergency treatment.

The term "small vessel disease" encompasses a range of conditions that affect the small blood vessels in the brain, including:

1. Cerebral amyloid angiopathy (CAA): A condition characterized by the accumulation of beta-amyloid peptides in the walls of small blood vessels, leading to vascular inflammation and degeneration.
2. Cerebral infarction (CI): A condition caused by a blockage or rupture of small blood vessels in the brain, resulting in tissue damage or death due to lack of oxygen and nutrients.
3. Leukoaraiosis: A condition characterized by the degeneration of white matter in the brain, leading to a loss of myelin and axonal damage.
4. Moyamoya disease (MMD): A rare condition caused by stenosis or occlusion of the internal carotid artery and its branches, leading to decreased blood flow to the brain.
5. Small vessel ischemic change (SVIC): A condition characterized by the degeneration of small blood vessels in the brain due to chronic hypoperfusion or other factors.
6. Vasculitis: An inflammatory condition affecting the blood vessels in the brain, leading to damage and scarring.
7. Other conditions such as hypertension, diabetes, and hyperlipidemia can also contribute to the development of CSVD.

The exact pathophysiology of CSVD is complex and involves multiple factors, including genetic predisposition, aging, inflammation, oxidative stress, and vascular damage. The symptoms of CSVD can vary depending on the location and severity of the affected blood vessels, but may include cognitive decline, memory loss, difficulty with speech and language, weakness or numbness in the limbs, and vision problems.

CSVD is often difficult to diagnose, as its symptoms can be similar to other conditions such as Alzheimer's disease or stroke. A comprehensive diagnostic workup may include a physical examination, medical history, neuroimaging studies (such as CT or MRI scans), and laboratory tests to rule out other conditions.

There is currently no cure for CSVD, but various treatment options are available to manage its symptoms and slow its progression. These may include medications to control hypertension, diabetes, and hyperlipidemia; lifestyle modifications such as regular exercise and a healthy diet; and rehabilitation therapies to improve cognitive and motor function. In severe cases, surgical interventions such as bypass surgery or endarterectomy may be necessary.

In conclusion, CSVD is a complex and multifactorial condition that affects the blood vessels in the brain, leading to a range of cognitive and motor symptoms. While there is currently no cure for CSVD, various treatment options are available to manage its symptoms and slow its progression. Early detection and management of underlying risk factors can help to slow the progression of CSVD and improve outcomes for affected individuals.

Symptoms of intracranial hypertension can include headache, nausea and vomiting, confusion, seizures, and loss of consciousness. Treatment options depend on the underlying cause, but may include medications to reduce pressure, draining excess CSF, or surgery to relieve obstruction.

Intracranial hypertension can be life-threatening if left untreated, as it can lead to permanent brain damage and even death. Therefore, prompt medical attention is essential for proper diagnosis and management of this condition.

Hemiplegia can cause a range of symptoms including weakness, paralysis, loss of sensation, and difficulty with movement and coordination on one side of the body. The affected side may also experience muscle spasticity or rigidity, causing stiffness and limited mobility.

Depending on the severity and location of the damage, hemiplegia can be classified into different types:

1. Left hemiplegia: This type affects the left side of the body and is caused by damage to the left hemisphere of the brain.
2. Right hemiplegia: This type affects the right side of the body and is caused by damage to the right hemisphere of the brain.
3. Mixed hemiplegia: This type affects both sides of the body and is caused by damage to both hemispheres of the brain or other areas of the brainstem.
4. Progressive hemiplegia: This type progressively worsens over time and is often associated with neurodegenerative disorders such as Parkinson's disease or multiple sclerosis.

Treatment for hemiplegia typically focuses on physical therapy, occupational therapy, and rehabilitation to improve mobility, strength, and function. Medications such as anticonvulsants, muscle relaxants, and pain relievers may also be prescribed to manage symptoms. In severe cases, surgery may be necessary to relieve pressure on the brain or spinal cord.

In summary, hemiplegia is a condition characterized by paralysis or weakness on one side of the body, often caused by damage to the brain or spinal cord. Treatment options vary depending on the severity and underlying cause of the condition.

Anterior cerebral artery infarction is relatively rare compared to other types of strokes, but it tends to affect younger people more frequently than other types of strokes. The symptoms of anterior cerebral artery infarction can vary depending on the location and severity of the obstruction, but may include sudden weakness or numbness in one side of the body, difficulty speaking or understanding speech, confusion, and vision loss.

Prompt medical attention is essential for individuals experiencing these symptoms, as timely treatment can help to minimize damage to the affected brain tissue and improve outcomes. Treatment options for anterior cerebral artery infarction may include clot-busting drugs or mechanical thrombectomy, which involves removing the obstructive clot from the affected blood vessel. In some cases, surgery may also be necessary to relieve pressure on the affected blood vessels or to repair any damaged blood vessels.

Preventive measures for anterior cerebral artery infarction include controlling risk factors such as high blood pressure, diabetes, and high cholesterol levels, as well as avoiding activities that increase the risk of stroke such as smoking and heavy alcohol consumption. Early detection and treatment of any underlying medical conditions can help to reduce the risk of developing anterior cerebral artery infarction.

There are two main types of carotid stenosis:

1. Internal carotid artery stenosis: This type of stenosis occurs when the internal carotid artery, which supplies blood to the brain, becomes narrowed or blocked.
2. Common carotid artery stenosis: This type of stenosis occurs when the common carotid artery, which supplies blood to the head and neck, becomes narrowed or blocked.

The symptoms of carotid stenosis can vary depending on the severity of the blockage and the extent of the affected area. Some common symptoms include:

* Dizziness or lightheadedness
* Vertigo (a feeling of spinning)
* Blurred vision or double vision
* Memory loss or confusion
* Slurred speech
* Weakness or numbness in the face, arm, or leg on one side of the body

If left untreated, carotid stenosis can lead to a stroke or other serious complications. Treatment options for carotid stenosis include medications to lower cholesterol and blood pressure, as well as surgical procedures such as endarterectomy (removing plaque from the artery) or stenting (placing a small mesh tube in the artery to keep it open).

In conclusion, carotid stenosis is a serious medical condition that can lead to stroke and other complications if left untreated. It is important to seek medical attention if symptoms persist or worsen over time.

When the body's CO2 levels are too low, it can cause a range of symptoms including:

1. Dizziness and lightheadedness
2. Headaches
3. Fatigue and weakness
4. Confusion and disorientation
5. Numbness or tingling in the hands and feet
6. Muscle twitching
7. Irritability and anxiety
8. Increased heart rate and blood pressure
9. Sleep disturbances
10. Decreased mental performance and concentration

Hypocapnia can be diagnosed through a series of tests, including blood gas analysis, electroencephalography (EEG), and imaging studies such as computed tomography (CT) or magnetic resonance imaging (MRI). Treatment options vary depending on the underlying cause of hypocapnia, but may include breathing exercises, oxygen therapy, medication, and addressing any underlying conditions.

In severe cases, hypocapnia can lead to seizures, coma, and even death. Therefore, it is important to seek medical attention if symptoms persist or worsen over time.

The effects of hypoxia-ischemia on the brain can vary depending on the severity and duration of the insult, but may include:

* Cellular damage and death
* Inflammation and oxidative stress
* Neurotransmitter imbalances
* Blood-brain barrier disruption
* White matter degeneration

The long-term consequences of hypoxia-ischemia, brain may include cognitive impairments such as memory loss, attention deficits, and language difficulties. Behavioral changes, such as depression, anxiety, and mood swings, may also occur. In severe cases, the condition can lead to permanent vegetative state or death.

The diagnosis of hypoxia-ischemia, brain is based on a combination of clinical evaluation, laboratory tests, and imaging studies such as CT or MRI scans. Treatment options may include supportive care, medications, and rehabilitation therapies to address cognitive and behavioral impairments. In some cases, surgical interventions may be necessary to relieve pressure or restore blood flow to the affected areas.

Overall, hypoxia-ischemia, brain is a serious medical condition that requires prompt recognition and appropriate treatment to minimize long-term cognitive and functional impairments.

Sources:

1. American Heart Association. (n.d.). Cerebral Infarction (Brain Attack). Retrieved from
2. Mayo Clinic. (n.d.). Posterior cerebral artery infarction. Retrieved from
3. MedlinePlus. (n.d.). Posterior cerebral artery infarction. Retrieved from

In medical terminology, coma is defined as a state of prolonged unconsciousness that lasts for more than 24 hours and is characterized by a lack of responsiveness to stimuli, including pain, light, sound, or touch. Coma can be caused by a variety of factors, such as:

1. Traumatic brain injury: Coma can result from a severe head injury that causes damage to the brain.
2. Stroke: A stroke can cause coma if it affects a large part of the brain.
3. Infections: Bacterial or viral infections can spread to the brain and cause coma.
4. Poisoning: Toxic substances, such as drugs or chemicals, can cause coma by damaging the brain.
5. Hypoxia: Lack of oxygen to the brain can cause coma.
6. Hypoglycemia: Low blood sugar can cause coma.
7. Metabolic disorders: Certain metabolic disorders, such as diabetic ketoacidosis or hypothyroidism, can cause coma.
8. Electrolyte imbalance: An imbalance of electrolytes, such as sodium or potassium, can cause coma.
9. Chronic conditions: Certain chronic conditions, such as brain tumors or degenerative diseases like Alzheimer's or Parkinson's, can cause coma over time.

It is important to note that a coma is different from a vegetative state, which is characterized by awakening and opening one's eyes but lacking any meaningful response to stimuli. A comatose patient may also exhibit automatic responses, such as breathing or reacting to pain, but they are not aware of their surroundings or able to communicate.

The diagnosis of coma is typically made by a neurologist based on the patient's medical history, physical examination, and results of diagnostic tests such as electroencephalography (EEG) or imaging studies like computed tomography (CT) or magnetic resonance imaging (MRI). Treatment of coma depends on the underlying cause and may include supportive care, medication, or surgical intervention.

The symptoms of moyamoya disease typically begin in childhood or adolescence and can include:

* Recurring transient ischemic attacks (TIA, or "mini-strokes")
* Stroke or cerebral infarction
* Seizures
* Cognitive impairment or developmental delays
* Weakness or paralysis of the limbs
* Vision problems or blindness

The disease is caused by a combination of genetic and environmental factors, including:

* Genetic mutations that affect the formation and maintenance of blood vessels
* Environmental factors such as infections, trauma, or exposure to toxins

Moyamoya disease can be diagnosed through a variety of imaging tests, including:

* Computed tomography (CT) scans
* Magnetic resonance imaging (MRI)
* Magnetic resonance angiography (MRA)
* Positron emission tomography (PET) scans

There is no cure for moyamoya disease, but various treatments can be used to manage its symptoms and slow its progression. These may include:

* Medications to prevent or treat seizures, high blood pressure, or other complications
* Surgical procedures to improve blood flow to the brain, such as direct revascularization or bypass surgery
* Rehabilitation therapies to help regain lost function and mobility

Early diagnosis and treatment of moyamoya disease can help manage its symptoms and improve quality of life for affected individuals. However, because the disease is so rare and complex, it can be challenging to diagnose and treat effectively.

Some common causes of chronic brain damage include:

1. Traumatic brain injury (TBI): A blow to the head or other traumatic injury that causes the brain to bounce or twist inside the skull, leading to damage to brain cells and tissues.
2. Stroke or cerebral vasculature disorders: A loss of blood flow to the brain due to a blockage or rupture of blood vessels, leading to cell death and tissue damage.
3. Infections such as meningitis or encephalitis: Inflammation of the brain and its membranes caused by viral or bacterial infections, which can lead to damage to brain cells and tissues.
4. Chronic exposure to toxins, such as pesticides or heavy metals: Prolonged exposure to these substances can damage brain cells and tissues over time.
5. Neurodegenerative diseases, such as Alzheimer's disease or Parkinson's disease: These conditions are characterized by the progressive loss of brain cells and tissue, leading to cognitive decline and other symptoms.

The effects of chronic brain damage can vary depending on the location and severity of the damage. Some common effects include:

1. Cognitive impairments: Difficulty with memory, attention, problem-solving, and other cognitive functions.
2. Emotional and behavioral changes: Depression, anxiety, irritability, and mood swings.
3. Physical symptoms: Weakness or paralysis on one side of the body, difficulty with balance and coordination, and changes in sensation or perception.
4. Communication difficulties: Slurred speech, difficulty finding the right words, and trouble understanding spoken language.
5. Social and occupational impairments: Difficulty with daily activities, social interactions, and work-related tasks.

The good news is that there are several strategies that can help mitigate the effects of chronic brain damage. These include:

1. Physical exercise: Regular physical activity has been shown to promote brain health and reduce the risk of cognitive decline.
2. Cognitive stimulation: Engaging in mentally challenging activities, such as reading, puzzles, or learning a new skill, can help build cognitive reserve and reduce the risk of cognitive decline.
3. Social engagement: Building and maintaining social connections has been shown to promote brain health and reduce the risk of cognitive decline.
4. Stress management: Chronic stress can exacerbate brain damage, so finding ways to manage stress, such as through meditation or exercise, is important.
5. Proper nutrition: Eating a diet rich in fruits, vegetables, and omega-3 fatty acids can help support brain health and reduce the risk of cognitive decline.
6. Medication and therapy: In some cases, medication or therapy may be necessary to manage the symptoms of chronic brain damage.
7. Neuroplasticity-based interventions: Techniques that promote neuroplasticity, such as non-invasive brain stimulation, can help improve cognitive function and reduce the risk of cognitive decline.

It's important to note that these strategies may not reverse chronic brain damage, but they can help mitigate its effects and improve overall brain health. If you suspect that you or someone you know may be experiencing chronic brain damage, it is important to seek medical attention as soon as possible. Early diagnosis and treatment can help reduce the risk of long-term cognitive decline and improve quality of life.

Examples of acute diseases include:

1. Common cold and flu
2. Pneumonia and bronchitis
3. Appendicitis and other abdominal emergencies
4. Heart attacks and strokes
5. Asthma attacks and allergic reactions
6. Skin infections and cellulitis
7. Urinary tract infections
8. Sinusitis and meningitis
9. Gastroenteritis and food poisoning
10. Sprains, strains, and fractures.

Acute diseases can be treated effectively with antibiotics, medications, or other therapies. However, if left untreated, they can lead to chronic conditions or complications that may require long-term care. Therefore, it is important to seek medical attention promptly if symptoms persist or worsen over time.

There are several potential causes of hyperventilation, including anxiety, panic attacks, and certain medical conditions such as asthma or chronic obstructive pulmonary disease (COPD). Treatment for hyperventilation typically involves slowing down the breathing rate and restoring the body's natural balance of oxygen and carbon dioxide levels.

Some common signs and symptoms of hyperventilation include:

* Rapid breathing
* Deep breathing
* Dizziness or lightheadedness
* Chest pain or tightness
* Shortness of breath
* Confusion or disorientation
* Nausea or vomiting

If you suspect that someone is experiencing hyperventilation, it is important to seek medical attention immediately. Treatment may involve the following:

1. Oxygen therapy: Providing extra oxygen to help restore normal oxygen levels in the body.
2. Breathing exercises: Teaching the individual deep, slow breathing exercises to help regulate their breathing pattern.
3. Relaxation techniques: Encouraging the individual to relax and reduce stress, which can help slow down their breathing rate.
4. Medications: In severe cases, medications such as sedatives or anti-anxiety drugs may be prescribed to help calm the individual and regulate their breathing.
5. Ventilation support: In severe cases of hyperventilation, mechanical ventilation may be necessary to support the individual's breathing.

It is important to seek medical attention if you or someone you know is experiencing symptoms of hyperventilation, as it can lead to more serious complications such as respiratory failure or cardiac arrest if left untreated.

There are many different types of epilepsy, each with its own unique set of symptoms and characteristics. Some common forms of epilepsy include:

1. Generalized Epilepsy: This type of epilepsy affects both sides of the brain and can cause a range of seizure types, including absence seizures, tonic-clonic seizures, and atypical absence seizures.
2. Focal Epilepsy: This type of epilepsy affects only one part of the brain and can cause seizures that are localized to that area. There are several subtypes of focal epilepsy, including partial seizures with complex symptoms and simple partial seizures.
3. Tonic-Clonic Epilepsy: This type of epilepsy is also known as grand mal seizures and can cause a loss of consciousness, convulsions, and muscle stiffness.
4. Lennox-Gastaut Syndrome: This is a rare and severe form of epilepsy that typically develops in early childhood and can cause multiple types of seizures, including tonic, atonic, and myoclonic seizures.
5. Dravet Syndrome: This is a rare genetic form of epilepsy that typically develops in infancy and can cause severe, frequent seizures.
6. Rubinstein-Taybi Syndrome: This is a rare genetic disorder that can cause intellectual disability, developmental delays, and various types of seizures.
7. Other forms of epilepsy include Absence Epilepsy, Myoclonic Epilepsy, and Atonic Epilepsy.

The symptoms of epilepsy can vary widely depending on the type of seizure disorder and the individual affected. Some common symptoms of epilepsy include:

1. Seizures: This is the most obvious symptom of epilepsy and can range from mild to severe.
2. Loss of consciousness: Some people with epilepsy may experience a loss of consciousness during a seizure, while others may remain aware of their surroundings.
3. Confusion and disorientation: After a seizure, some people with epilepsy may feel confused and disoriented.
4. Memory loss: Seizures can cause short-term or long-term memory loss.
5. Fatigue: Epilepsy can cause extreme fatigue, both during and after a seizure.
6. Emotional changes: Some people with epilepsy may experience emotional changes, such as anxiety, depression, or mood swings.
7. Cognitive changes: Epilepsy can affect cognitive function, including attention, memory, and learning.
8. Sleep disturbances: Some people with epilepsy may experience sleep disturbances, such as insomnia or sleepiness.
9. Physical symptoms: Depending on the type of seizure, people with epilepsy may experience physical symptoms such as muscle weakness, numbness or tingling, and sensory changes.
10. Social isolation: Epilepsy can cause social isolation due to fear of having a seizure in public or stigma associated with the condition.

It's important to note that not everyone with epilepsy will experience all of these symptoms, and some people may have different symptoms depending on the type of seizure they experience. Additionally, some people with epilepsy may experience additional symptoms not listed here.

The most common carotid artery disease is atherosclerosis, which is the buildup of plaque in the inner lining of the arteries. This buildup can lead to a narrowing or blockage of the arteries, reducing blood flow to the brain and increasing the risk of stroke. Other conditions that can affect the carotid arteries include:

1. Carotid artery stenosis: A narrowing of the carotid arteries caused by atherosclerosis or other factors.
2. Carotid artery dissection: A tear in the inner lining of the arteries that can cause bleeding and blockage.
3. Carotid artery aneurysm: A bulge in the wall of the arteries that can lead to rupture and stroke.
4. Temporal bone fracture: A break in the bones of the skull that can cause damage to the carotid arteries and result in stroke or other complications.

Carotid artery diseases are typically diagnosed using imaging tests such as ultrasound, computed tomography (CT) angiography, or magnetic resonance angiography (MRA). Treatment options for carotid artery diseases depend on the underlying condition and its severity, but may include lifestyle changes, medications, surgery, or endovascular procedures.

Prevention of carotid artery diseases is key to reducing the risk of stroke and other complications. This includes managing risk factors such as high blood pressure, high cholesterol, smoking, and diabetes, as well as maintaining a healthy lifestyle and getting regular check-ups with your doctor.

Brain neoplasms can arise from various types of cells in the brain, including glial cells (such as astrocytes and oligodendrocytes), neurons, and vascular tissues. The symptoms of brain neoplasms vary depending on their size, location, and type, but may include headaches, seizures, weakness or numbness in the limbs, and changes in personality or cognitive function.

There are several different types of brain neoplasms, including:

1. Meningiomas: These are benign tumors that arise from the meninges, the thin layers of tissue that cover the brain and spinal cord.
2. Gliomas: These are malignant tumors that arise from glial cells in the brain. The most common type of glioma is a glioblastoma, which is aggressive and hard to treat.
3. Pineal parenchymal tumors: These are rare tumors that arise in the pineal gland, a small endocrine gland in the brain.
4. Craniopharyngiomas: These are benign tumors that arise from the epithelial cells of the pituitary gland and the hypothalamus.
5. Medulloblastomas: These are malignant tumors that arise in the cerebellum, specifically in the medulla oblongata. They are most common in children.
6. Acoustic neurinomas: These are benign tumors that arise on the nerve that connects the inner ear to the brain.
7. Oligodendrogliomas: These are malignant tumors that arise from oligodendrocytes, the cells that produce the fatty substance called myelin that insulates nerve fibers.
8. Lymphomas: These are cancers of the immune system that can arise in the brain and spinal cord. The most common type of lymphoma in the CNS is primary central nervous system (CNS) lymphoma, which is usually a type of B-cell non-Hodgkin lymphoma.
9. Metastatic tumors: These are tumors that have spread to the brain from another part of the body. The most common types of metastatic tumors in the CNS are breast cancer, lung cancer, and melanoma.

These are just a few examples of the many types of brain and spinal cord tumors that can occur. Each type of tumor has its own unique characteristics, such as its location, size, growth rate, and biological behavior. These factors can help doctors determine the best course of treatment for each patient.

Examples of Nervous System Diseases include:

1. Alzheimer's disease: A progressive neurological disorder that affects memory and cognitive function.
2. Parkinson's disease: A degenerative disorder that affects movement, balance and coordination.
3. Multiple sclerosis: An autoimmune disease that affects the protective covering of nerve fibers.
4. Stroke: A condition where blood flow to the brain is interrupted, leading to brain cell death.
5. Brain tumors: Abnormal growth of tissue in the brain.
6. Neuropathy: Damage to peripheral nerves that can cause pain, numbness and weakness in hands and feet.
7. Epilepsy: A disorder characterized by recurrent seizures.
8. Motor neuron disease: Diseases that affect the nerve cells responsible for controlling voluntary muscle movement.
9. Chronic pain syndrome: Persistent pain that lasts more than 3 months.
10. Neurodevelopmental disorders: Conditions such as autism, ADHD and learning disabilities that affect the development of the brain and nervous system.

These diseases can be caused by a variety of factors such as genetics, infections, injuries, toxins and ageing. Treatment options for Nervous System Diseases range from medications, surgery, rehabilitation therapy to lifestyle changes.

There are several causes of hypotension, including:

1. Dehydration: Loss of fluids and electrolytes can cause a drop in blood pressure.
2. Blood loss: Losing too much blood can lead to hypotension.
3. Medications: Certain medications, such as diuretics and beta-blockers, can lower blood pressure.
4. Heart conditions: Heart failure, cardiac tamponade, and arrhythmias can all cause hypotension.
5. Endocrine disorders: Hypothyroidism (underactive thyroid) and adrenal insufficiency can cause low blood pressure.
6. Vasodilation: A condition where the blood vessels are dilated, leading to low blood pressure.
7. Sepsis: Severe infection can cause hypotension.

Symptoms of hypotension can include:

1. Dizziness and lightheadedness
2. Fainting or passing out
3. Weakness and fatigue
4. Confusion and disorientation
5. Pale, cool, or clammy skin
6. Fast or weak pulse
7. Shortness of breath
8. Nausea and vomiting

If you suspect that you or someone else is experiencing hypotension, it is important to seek medical attention immediately. Treatment will depend on the underlying cause of the condition, but may include fluids, electrolytes, and medication to raise blood pressure. In severe cases, hospitalization may be necessary.

A blockage caused by air bubbles in the bloodstream, which can occur after a sudden change in atmospheric pressure (e.g., during an airplane flight or scuba diving). Air embolism can cause a variety of symptoms, including shortness of breath, chest pain, and stroke. It is a potentially life-threatening condition that requires prompt medical attention.

Note: Air embolism can also occur in the venous system, causing a pulmonary embolism (blockage of an artery in the lungs). This is a more common condition and is discussed separately.

There are several types of hydrocephalus, including:

1. Aqueductal stenosis: This occurs when the aqueduct that connects the third and fourth ventricles becomes narrowed or blocked, leading to an accumulation of CSF in the brain.
2. Choroid plexus papilloma: This is a benign tumor that grows on the surface of the choroid plexus, which is a layer of tissue that produces CSF.
3. Hydrocephalus ex vacuo: This occurs when there is a decrease in the volume of brain tissue due to injury or disease, leading to an accumulation of CSF.
4. Normal pressure hydrocephalus (NPH): This is a type of hydrocephalus that occurs in adults and is characterized by an enlarged ventricle, gait disturbances, and cognitive decline, despite normal pressure levels.
5. Symptomatic hydrocephalus: This type of hydrocephalus is caused by other conditions such as brain tumors, cysts, or injuries.

Symptoms of hydrocephalus can include headache, nausea, vomiting, seizures, and difficulty walking or speaking. Treatment options for hydrocephalus depend on the underlying cause and may include medication, surgery, or a shunt to drain excess CSF. In some cases, hydrocephalus can be managed with lifestyle modifications such as regular exercise and a balanced diet.

Prognosis for hydrocephalus varies depending on the underlying cause and severity of the condition. However, with timely diagnosis and appropriate treatment, many people with hydrocephalus can lead active and fulfilling lives.

Hemangiomas are caused by an abnormal formation of blood vessels during fetal development. They are more common in infants and children, but they can also occur in adults. The exact cause of CNS hemangiomas is not fully understood, but genetic mutations, environmental factors, and hormonal influences have been implicated.

The symptoms of CNS hemangiomas can vary depending on their location and size. Large hemangiomas can cause pressure on surrounding brain tissue, leading to symptoms such as headaches, seizures, and developmental delays. Smaller hemangiomas may not cause any symptoms at all, but they can still be detected through imaging tests such as MRI or CT scans.

Hemangiomas can occur anywhere in the CNS, but they are most commonly found in the brain, specifically in the cerebral cortex and basal ganglia. They can also occur in the spinal cord, where they can cause symptoms such as pain, numbness, and weakness in the limbs.

The diagnosis of a CNS hemangioma is based on a combination of clinical findings, imaging studies, and histopathological analysis. Imaging studies, such as MRI or CT scans, can help identify the location and size of the hemangioma, while histopathological analysis can confirm the presence of dilated blood vessels.

There is no specific treatment for CNS hemangiomas, but various options are available depending on the severity of the condition and the symptoms it causes. Observation, corticosteroids, and surgery are some of the most common treatments used to manage CNS hemangiomas. In some cases, interventional techniques such as embolization or stereotactic radiosurgery may be necessary to treat the condition.

Overall, CNS hemangiomas are benign vascular tumors that can cause a range of symptoms and cognitive impairments in children and adults. While there is no specific treatment for these tumors, various options are available to manage their symptoms and improve quality of life. It is important to seek medical attention if symptoms persist or worsen over time, as early diagnosis and treatment can significantly improve outcomes.

There are several types of atrophy that can occur in different parts of the body. For example:

1. Muscular atrophy: This occurs when muscles weaken and shrink due to disuse or injury.
2. Neuronal atrophy: This occurs when nerve cells degenerate, leading to a loss of cognitive function and memory.
3. Cardiac atrophy: This occurs when the heart muscle weakens and becomes less efficient, leading to decreased cardiac output.
4. Atrophic gastritis: This is a type of stomach inflammation that can lead to the wasting away of the stomach lining.
5. Atrophy of the testes: This occurs when the testes shrink due to a lack of use or disorder, leading to decreased fertility.

Atrophy can be diagnosed through various medical tests and imaging studies, such as MRI or CT scans. Treatment for atrophy depends on the underlying cause and may involve physical therapy, medication, or surgery. In some cases, atrophy can be prevented or reversed with proper treatment and care.

In summary, atrophy is a degenerative process that can occur in various parts of the body due to injury, disease, or disuse. It can lead to a loss of function and decreased quality of life, but with proper diagnosis and treatment, it may be possible to prevent or reverse some forms of atrophy.

The symptoms of Alzheimer's disease can vary from person to person and may progress slowly over time. Early symptoms may include memory loss, confusion, and difficulty with problem-solving. As the disease progresses, individuals may experience language difficulties, visual hallucinations, and changes in mood and behavior.

There is currently no cure for Alzheimer's disease, but there are several medications and therapies that can help manage its symptoms and slow its progression. These include cholinesterase inhibitors, memantine, and non-pharmacological interventions such as cognitive training and behavioral therapy.

Alzheimer's disease is a significant public health concern, affecting an estimated 5.8 million Americans in 2020. It is the sixth leading cause of death in the United States, and its prevalence is expected to continue to increase as the population ages.

There is ongoing research into the causes and potential treatments for Alzheimer's disease, including studies into the role of inflammation, oxidative stress, and the immune system. Other areas of research include the development of biomarkers for early detection and the use of advanced imaging techniques to monitor progression of the disease.

Overall, Alzheimer's disease is a complex and multifactorial disorder that poses significant challenges for individuals, families, and healthcare systems. However, with ongoing research and advances in medical technology, there is hope for improving diagnosis and treatment options in the future.

The symptoms of a brain abscess can vary depending on the location and size of the abscess, but may include:

* Headache
* Fever
* Confusion or disorientation
* Seizures
* Weakness or numbness in the arms or legs
* Vision problems
* Speech difficulties

If a brain abscess is suspected, a doctor will typically perform a physical examination and order imaging tests such as CT or MRI scans to confirm the diagnosis. Treatment usually involves antibiotics to treat the underlying infection, as well as surgery to drain the abscess and remove any infected tissue. In severe cases, hospitalization may be necessary to monitor and treat the patient.

With prompt and appropriate treatment, most people with a brain abscess can recover fully or almost fully, but in some cases, the condition can result in long-term complications such as memory loss, cognitive impairment, or personality changes. In rare instances, a brain abscess can be fatal if not treated promptly and properly.

There are different types of anoxia, including:

1. Cerebral anoxia: This occurs when the brain does not receive enough oxygen, leading to cognitive impairment, confusion, and loss of consciousness.
2. Pulmonary anoxia: This occurs when the lungs do not receive enough oxygen, leading to shortness of breath, coughing, and chest pain.
3. Cardiac anoxia: This occurs when the heart does not receive enough oxygen, leading to cardiac arrest and potentially death.
4. Global anoxia: This is a complete lack of oxygen to the entire body, leading to widespread tissue damage and death.

Treatment for anoxia depends on the underlying cause and the severity of the condition. In some cases, hospitalization may be necessary to provide oxygen therapy, pain management, and other supportive care. In severe cases, anoxia can lead to long-term disability or death.

Prevention of anoxia is important, and this includes managing underlying medical conditions such as heart disease, diabetes, and respiratory problems. It also involves avoiding activities that can lead to oxygen deprivation, such as scuba diving or high-altitude climbing, without proper training and equipment.

In summary, anoxia is a serious medical condition that occurs when there is a lack of oxygen in the body or specific tissues or organs. It can cause cell death and tissue damage, leading to serious health complications and even death if left untreated. Early diagnosis and treatment are crucial to prevent long-term disability or death.

Symptoms include:

* Sudden loss of vision in one eye
* Blind spot or dark area in the visual field
* No pain or discomfort

Diagnosis is typically made through a comprehensive eye exam, including visual acuity testing, dilated eye exam, and imaging tests such as fluorescein angiography.

Treatment for amaurosis fugax depends on the underlying cause, but may include:

* Medications to improve blood flow and reduce inflammation
* Laser or surgical procedures to improve blood flow or remove blockages
* Monitoring of blood pressure and cholesterol levels to prevent future episodes

It is important to seek medical attention if you experience a sudden loss of vision, as prompt treatment can help to prevent long-term damage and improve the chance of recovery.

There are several types of intracranial hemorrhage, including:

1. Cerebral hemorrhage: Bleeding within the cerebral tissue itself, which can cause damage to brain cells and lead to a variety of complications.
2. Subarachnoid hemorrhage: Bleeding between the brain and the thin membrane that covers it (the meninges), which can cause severe headaches and other symptoms.
3. Epidural hemorrhage: Bleeding between the dura mater, a protective layer of tissue surrounding the brain, and the skull.
4. Subdural hemorrhage: Bleeding between the dura mater and the arachnoid membrane, which can cause severe headaches and other symptoms.

The symptoms of intracranial hemorrhage can vary depending on the location and severity of the bleeding, but may include:

* Sudden, severe headache
* Nausea and vomiting
* Confusion and disorientation
* Weakness or numbness in the face, arm, or leg
* Seizures
* Loss of consciousness

Diagnosis is typically made through a combination of physical examination, imaging tests (such as CT or MRI scans), and laboratory tests to determine the cause of the hemorrhage. Treatment depends on the location and severity of the bleeding, but may include medications to control symptoms, surgery to repair the source of the bleeding, or other interventions as needed.

Polyploidy is a condition where an organism has more than two sets of chromosomes, which are the thread-like structures that carry genetic information. It can occur in both plants and animals, although it is relatively rare in most species. In humans, polyploidy is extremely rare and usually occurs as a result of errors during cell division or abnormal fertilization.

In medicine, polyploidy is often used to describe certain types of cancer, such as breast cancer or colon cancer, that have extra sets of chromosomes. This can lead to the development of more aggressive and difficult-to-treat tumors.

However, not all cases of polyploidy are cancerous. Some individuals with Down syndrome, for example, have an extra copy of chromosome 21, which is a non-cancerous form of polyploidy. Additionally, some people may be born with extra copies of certain genes or chromosomal regions due to errors during embryonic development, which can lead to various health problems but are not cancerous.

Overall, the term "polyploidy" in medicine is used to describe any condition where an organism has more than two sets of chromosomes, regardless of whether it is cancerous or non-cancerous.

PVL is often seen in premature infants, especially those born before 32 weeks of gestation, as their brains are not fully developed and are more susceptible to injury. It can also occur in full-term newborns who have experienced hypoxia (lack of oxygen) during delivery or shortly after birth.

The symptoms of PVL can vary depending on the severity of the condition and may include:

* Delayed developmental milestones
* Poor muscle tone and coordination
* Seizures
* Vision problems
* Hearing loss

PVL is typically diagnosed through a combination of physical examination, medical history, and imaging studies such as ultrasound or MRI. Treatment for PVL often focuses on managing the underlying cause, such as hypoxia or infection, and providing supportive care to help the brain heal. In some cases, medications may be prescribed to help control seizures or other symptoms.

Overall, periventricular leukomalacia is a serious condition that can have long-lasting effects on the developing brain, but with proper medical care and support, many children are able to recover and lead normal lives.

There are many different types of seizures, each with its own unique set of symptoms. Some common types of seizures include:

1. Generalized seizures: These seizures affect both sides of the brain and can cause a range of symptoms, including convulsions, loss of consciousness, and muscle stiffness.
2. Focal seizures: These seizures affect only one part of the brain and can cause more specific symptoms, such as weakness or numbness in a limb, or changes in sensation or vision.
3. Tonic-clonic seizures: These seizures are also known as grand mal seizures and can cause convulsions, loss of consciousness, and muscle stiffness.
4. Absence seizures: These seizures are also known as petit mal seizures and can cause a brief loss of consciousness or staring spell.
5. Myoclonic seizures: These seizures can cause sudden, brief muscle jerks or twitches.
6. Atonic seizures: These seizures can cause a sudden loss of muscle tone, which can lead to falls or drops.
7. Lennox-Gastaut syndrome: This is a rare and severe form of epilepsy that can cause multiple types of seizures, including tonic, atonic, and myoclonic seizures.

Seizures can be diagnosed through a combination of medical history, physical examination, and diagnostic tests such as electroencephalography (EEG) or imaging studies. Treatment for seizures usually involves anticonvulsant medications, but in some cases, surgery or other interventions may be necessary.

Overall, seizures are a complex and multifaceted symptom that can have a significant impact on an individual's quality of life. It is important to seek medical attention if you or someone you know is experiencing seizures, as early diagnosis and treatment can help to improve outcomes and reduce the risk of complications.

Encephalitis can cause a range of symptoms, including fever, headache, confusion, seizures, and loss of consciousness. In severe cases, encephalitis can lead to brain damage, coma, and even death.

The diagnosis of encephalitis is based on a combination of clinical signs, laboratory tests, and imaging studies. Laboratory tests may include blood tests to detect the presence of antibodies or antigens specific to the causative agent, as well as cerebrospinal fluid (CSF) analysis to look for inflammatory markers and/or bacteria or viruses in the CSF. Imaging studies, such as CT or MRI scans, may be used to visualize the brain and identify any areas of damage or inflammation.

Treatment of encephalitis typically involves supportive care, such as intravenous fluids, oxygen therapy, and medication to manage fever and pain. Antiviral or antibacterial drugs may be used to target the specific causative agent, if identified. In severe cases, hospitalization in an intensive care unit (ICU) may be necessary to monitor and manage the patient's condition.

Prevention of encephalitis includes vaccination against certain viruses that can cause the condition, such as herpes simplex virus and Japanese encephalitis virus. Additionally, avoiding exposure to mosquitoes and other insects that can transmit viruses or bacteria that cause encephalitis, as well as practicing good hygiene and sanitation, can help reduce the risk of infection.

Overall, encephalitis is a serious and potentially life-threatening condition that requires prompt medical attention for proper diagnosis and treatment. With appropriate care, many patients with encephalitis can recover fully or partially, but some may experience long-term neurological complications or disability.

Types of Cognition Disorders: There are several types of cognitive disorders that affect different aspects of cognitive functioning. Some common types include:

1. Attention Deficit Hyperactivity Disorder (ADHD): Characterized by symptoms of inattention, hyperactivity, and impulsivity.
2. Traumatic Brain Injury (TBI): Caused by a blow or jolt to the head that disrupts brain function, resulting in cognitive, emotional, and behavioral changes.
3. Alzheimer's Disease: A progressive neurodegenerative disorder characterized by memory loss, confusion, and difficulty with communication.
4. Stroke: A condition where blood flow to the brain is interrupted, leading to cognitive impairment and other symptoms.
5. Parkinson's Disease: A neurodegenerative disorder that affects movement, balance, and cognition.
6. Huntington's Disease: An inherited disorder that causes progressive damage to the brain, leading to cognitive decline and other symptoms.
7. Frontotemporal Dementia (FTD): A group of neurodegenerative disorders characterized by changes in personality, behavior, and language.
8. Post-Traumatic Stress Disorder (PTSD): A condition that develops after a traumatic event, characterized by symptoms such as anxiety, avoidance, and hypervigilance.
9. Mild Cognitive Impairment (MCI): A condition characterized by memory loss and other cognitive symptoms that are more severe than normal age-related changes but not severe enough to interfere with daily life.

Causes and Risk Factors: The causes of cognition disorders can vary depending on the specific disorder, but some common risk factors include:

1. Genetics: Many cognitive disorders have a genetic component, such as Alzheimer's disease, Parkinson's disease, and Huntington's disease.
2. Age: As people age, their risk of developing cognitive disorders increases, such as Alzheimer's disease, vascular dementia, and frontotemporal dementia.
3. Lifestyle factors: Factors such as physical inactivity, smoking, and poor diet can increase the risk of cognitive decline and dementia.
4. Traumatic brain injury: A severe blow to the head or a traumatic brain injury can increase the risk of developing cognitive disorders, such as chronic traumatic encephalopathy (CTE).
5. Infections: Certain infections, such as meningitis and encephalitis, can cause cognitive disorders if they damage the brain tissue.
6. Stroke or other cardiovascular conditions: A stroke or other cardiovascular conditions can cause cognitive disorders by damaging the blood vessels in the brain.
7. Chronic substance abuse: Long-term use of drugs or alcohol can damage the brain and increase the risk of cognitive disorders, such as dementia.
8. Sleep disorders: Sleep disorders, such as sleep apnea, can increase the risk of cognitive disorders, such as dementia.
9. Depression and anxiety: Mental health conditions, such as depression and anxiety, can increase the risk of cognitive decline and dementia.
10. Environmental factors: Exposure to certain environmental toxins, such as pesticides and heavy metals, has been linked to an increased risk of cognitive disorders.

It's important to note that not everyone with these risk factors will develop a cognitive disorder, and some people without any known risk factors can still develop a cognitive disorder. If you have concerns about your cognitive health, it's important to speak with a healthcare professional for proper evaluation and diagnosis.

There are two types of hypertension:

1. Primary Hypertension: This type of hypertension has no identifiable cause and is also known as essential hypertension. It accounts for about 90% of all cases of hypertension.
2. Secondary Hypertension: This type of hypertension is caused by an underlying medical condition or medication. It accounts for about 10% of all cases of hypertension.

Some common causes of secondary hypertension include:

* Kidney disease
* Adrenal gland disorders
* Hormonal imbalances
* Certain medications
* Sleep apnea
* Cocaine use

There are also several risk factors for hypertension, including:

* Age (the risk increases with age)
* Family history of hypertension
* Obesity
* Lack of exercise
* High sodium intake
* Low potassium intake
* Stress

Hypertension is often asymptomatic, and it can cause damage to the blood vessels and organs over time. Some potential complications of hypertension include:

* Heart disease (e.g., heart attacks, heart failure)
* Stroke
* Kidney disease (e.g., chronic kidney disease, end-stage renal disease)
* Vision loss (e.g., retinopathy)
* Peripheral artery disease

Hypertension is typically diagnosed through blood pressure readings taken over a period of time. Treatment for hypertension may include lifestyle changes (e.g., diet, exercise, stress management), medications, or a combination of both. The goal of treatment is to reduce the risk of complications and improve quality of life.

Example sentences:

1. The patient was diagnosed with multi-infarct dementia after a series of small strokes were discovered on an MRI scan.
2. The risk of developing multi-infarct dementia increases with age and is more common in individuals with a history of cardiovascular disease.
3. Multi-infarct dementia can cause significant impairment in daily functioning and requires ongoing medical management to slow progression of the condition.

The symptoms of vascular dementia can vary depending on the location and severity of the damage to the brain, but common symptoms include:

* Memory loss, such as difficulty remembering recent events or learning new information
* Confusion and disorientation
* Difficulty with communication, including trouble finding the right words or understanding what others are saying
* Difficulty with problem-solving, decision-making, and judgment
* Mood changes, such as depression, anxiety, or agitation
* Personality changes, such as becoming more passive or suspicious
* Difficulty with coordination and movement, including trouble walking or balance

Vascular dementia can be caused by a variety of conditions that affect the blood vessels in the brain, including:

* Stroke or transient ischemic attack (TIA, or "mini-stroke")
* Small vessel disease, such as tiny strokes or changes in the blood vessels that occur over time
* Moyamoya disease, a rare condition caused by narrowing or blockage of the internal carotid artery and its branches
* Cerebral amyloid angiopathy, a condition in which abnormal protein deposits build up in the blood vessels of the brain
* Other conditions that can cause reduced blood flow to the brain, such as high blood pressure, diabetes, or cardiovascular disease

There is no cure for vascular dementia, but there are several treatment options available to help manage its symptoms and slow its progression. These may include medications to improve memory and cognitive function, physical therapy to maintain mobility and strength, and lifestyle changes such as a healthy diet and regular exercise. In some cases, surgery or endovascular procedures may be recommended to treat the underlying cause of the dementia, such as a stroke or blocked blood vessel.

It is important for individuals with vascular dementia to receive timely and accurate diagnosis and treatment, as well as ongoing support and care from healthcare professionals, family members, and caregivers. With appropriate management, many people with vascular dementia are able to maintain their independence and quality of life for as long as possible.

There are several types of headaches, including:

1. Tension headache: This is the most common type of headache and is caused by muscle tension in the neck and scalp.
2. Migraine: This is a severe headache that can cause nausea, vomiting, and sensitivity to light and sound.
3. Sinus headache: This type of headache is caused by inflammation or infection in the sinuses.
4. Cluster headache: This is a rare type of headache that occurs in clusters or cycles and can be very painful.
5. Rebound headache: This type of headache is caused by overuse of pain medication.

Headaches can be treated with a variety of methods, such as:

1. Over-the-counter pain medications, such as acetaminophen or ibuprofen.
2. Prescription medications, such as triptans or ergots, for migraines and other severe headaches.
3. Lifestyle changes, such as stress reduction techniques, regular exercise, and a healthy diet.
4. Alternative therapies, such as acupuncture or massage, which can help relieve tension and pain.
5. Addressing underlying causes, such as sinus infections or allergies, that may be contributing to the headaches.

It is important to seek medical attention if a headache is severe, persistent, or accompanied by other symptoms such as fever, confusion, or weakness. A healthcare professional can diagnose the cause of the headache and recommend appropriate treatment.

Body weight is an important health indicator, as it can affect an individual's risk for certain medical conditions, such as obesity, diabetes, and cardiovascular disease. Maintaining a healthy body weight is essential for overall health and well-being, and there are many ways to do so, including a balanced diet, regular exercise, and other lifestyle changes.

There are several ways to measure body weight, including:

1. Scale: This is the most common method of measuring body weight, and it involves standing on a scale that displays the individual's weight in kg or lb.
2. Body fat calipers: These are used to measure body fat percentage by pinching the skin at specific points on the body.
3. Skinfold measurements: This method involves measuring the thickness of the skin folds at specific points on the body to estimate body fat percentage.
4. Bioelectrical impedance analysis (BIA): This is a non-invasive method that uses electrical impulses to measure body fat percentage.
5. Dual-energy X-ray absorptiometry (DXA): This is a more accurate method of measuring body composition, including bone density and body fat percentage.

It's important to note that body weight can fluctuate throughout the day due to factors such as water retention, so it's best to measure body weight at the same time each day for the most accurate results. Additionally, it's important to use a reliable scale or measuring tool to ensure accurate measurements.

Muscle spasticity can cause a range of symptoms, including:

* Increased muscle tone, leading to stiffness and rigidity
* Spasms or sudden contractions of the affected muscles
* Difficulty moving the affected limbs
* Pain or discomfort in the affected area
* Abnormal postures or movements

There are several potential causes of muscle spasticity, including:

* Neurological disorders such as cerebral palsy, multiple sclerosis, and spinal cord injuries
* Stroke or other brain injuries
* Muscle damage or inflammation
* Infections such as meningitis or encephalitis
* Metabolic disorders such as hypokalemia (low potassium levels) or hyperthyroidism

Treatment options for muscle spasticity include:

* Physical therapy to improve range of motion and strength
* Medications such as baclofen, tizanidine, or dantrolene to reduce muscle spasms
* Injectable medications such as botulinum toxin or phenol to destroy excess nerve fibers
* Surgery to release or sever affected nerve fibers
* Electrical stimulation therapy to improve muscle function and reduce spasticity.

It is important to note that muscle spasticity can have a significant impact on an individual's quality of life, affecting their ability to perform daily activities, maintain independence, and engage in social and recreational activities. As such, it is important to seek medical attention if symptoms of muscle spasticity are present to determine the underlying cause and develop an appropriate treatment plan.

Carotid artery thrombosis is often caused by atherosclerosis, which is the buildup of plaque in the arteries that can lead to the formation of blood clots. Other risk factors for carotid artery thrombosis include high blood pressure, smoking, high cholesterol, diabetes, and obesity.

Diagnosis of carotid artery thrombosis typically involves imaging tests such as ultrasound, CT or MRI scans, and Doppler studies to visualize the blood flow in the neck and brain. Treatment options for carotid artery thrombosis include anticoagulation medications to prevent further clotting, medications to dissolve the clot, and surgery to remove the clot or repair the affected artery.

In severe cases, carotid artery thrombosis can lead to stroke or brain damage if not treated promptly. Therefore, it is important to seek medical attention immediately if symptoms persist or worsen over time.

* Cerebral encephalocele: when the brain tissue protrudes through the skull.
* Meningoencephalocele: when the meninges (the protective covering of the brain and spinal cord) protrude through the skull along with the brain tissue.
* Mesenchymal encephalocele: when other tissues such as skin, muscle or bone protrude through the skull along with the brain tissue.

Symptoms of encephalocele can vary depending on the severity of the defect and can include:

* Protrusion of the brain or meninges through a opening in the skull
* Abnormal appearance of the head or face
* Delayed developmental milestones such as sitting, standing or walking
* Poor muscle tone
* Seizures
* Vision and hearing problems

Diagnosis of encephalocele is typically made through a combination of physical examination, imaging studies such as CT or MRI scans, and genetic testing. Treatment for encephalocele usually involves surgery to repair the opening in the skull and relieve any pressure on the brain. In some cases, additional surgeries may be necessary to correct other defects such as hydrocephalus (fluid accumulation in the brain).

Encephalocele is a rare condition, but it can have serious consequences if left untreated. Early detection and intervention are important for improving outcomes and reducing the risk of complications.

In medical terms, craniocerebral trauma is defined as any injury that affects the skull, brain, or both, as a result of an external force. This can include fractures of the skull, intracranial hemorrhages (bleeding inside the skull), and diffuse axonal injuries (DAI), which are tears in the fibers of the brain.

Craniocerebral trauma can be classified into two main categories: closed head injury and open head injury. Closed head injury occurs when the skull does not fracture, but the brain is still affected by the impact, such as from whiplash or shaking. Open head injury, on the other hand, involves a fracture of the skull, which can cause the brain to be exposed to the outside environment and increase the risk of infection.

Treatment for craniocerebral trauma depends on the severity of the injury and may include observation, medication, surgery, or a combination of these. In severe cases, craniocerebral trauma can lead to long-term cognitive, emotional, and physical impairments, and may require ongoing rehabilitation and support.

The signs and symptoms of fetal hypoxia may include:

1. Decreased fetal movement
2. Abnormal fetal heart rate
3. Meconium staining of the amniotic fluid
4. Premature contractions
5. Preterm labor

If left untreated, fetal hypoxia can lead to serious complications such as:

1. Intracranial hemorrhage
2. Cerebral palsy
3. Developmental delays
4. Learning disabilities
5. Memory and cognitive impairments
6. Behavioral problems
7. Autism
8. Seizures
9. Hearing and vision loss

Treatment of fetal hypoxia depends on the underlying cause, but may include:

1. Bed rest or hospitalization
2. Corticosteroids to promote fetal growth and maturity
3. Oxygen supplementation
4. Antibiotics for infections
5. Planned delivery, if necessary

In some cases, fetal hypoxia may be detected through ultrasound examination, which can show a decrease in fetal movement or abnormal heart rate. However, not all cases of fetal hypoxia can be detected by ultrasound, and regular prenatal check-ups are essential to monitor the health of the developing fetus.

Prevention of fetal hypoxia includes proper prenatal care, avoiding harmful substances such as tobacco and alcohol, maintaining a healthy diet, and managing any underlying medical conditions. Early detection and treatment of fetal hypoxia can significantly improve outcomes for both the mother and the baby.

There are several types of strabismus, including:

* Esotropia: where one eye turns inward toward the nose
* Exotropia: where one eye turns outward away from the face
* Hypertropia: where one eye turns upward
* Hypotropia: where one eye turns downward
* Duane's syndrome: a rare type of strabismus that affects only one eye and is caused by nerve damage.

Strabismus can have both visual and social consequences, including:

* Difficulty with depth perception and binocular vision
* Blurred or double vision
* Difficulty with eye teaming and tracking
* Poor eye-hand coordination
* Social and emotional effects such as low self-esteem, anxiety, and depression.

Treatment options for strabismus include:

* Glasses or contact lenses to correct refractive errors
* Prism lenses to align the eyes
* Eye exercises to strengthen the muscles and improve eye teaming
* Surgery to adjust the position of the muscles that control eye movement.

It is important for individuals with strabismus to receive timely and appropriate treatment to address the underlying cause of the condition and prevent long-term vision loss and social difficulties.

The amblyopic eye may have reduced visual sharpness and/or abnormal ocular alignment (strabismus). The other eye is generally normal or has better vision. Amblyopia is often present at birth but may not be noticed until the child is a few years old. It can also result from various conditions, such as strabismus, cataracts, or differences in the refractive error of the two eyes (anisometropic amblyopia).

The most common form of amblyopia is anisometropic amblyopia, which occurs when there is a significant difference in the refractive power between the two eyes. This can cause the brain to favor one eye over the other, leading to reduced vision in the amblyopic eye. Amblyopia can be treated with glasses or contact lenses, patching the better eye to force the weaker eye to work harder, or surgery to correct strabismus or anisometropia.

Early detection and treatment are important to prevent long-term visual impairment. However, amblyopia can sometimes persist even after treatment, and it is a leading cause of monocular vision in adults.

Disease progression can be classified into several types based on the pattern of worsening:

1. Chronic progressive disease: In this type, the disease worsens steadily over time, with a gradual increase in symptoms and decline in function. Examples include rheumatoid arthritis, osteoarthritis, and Parkinson's disease.
2. Acute progressive disease: This type of disease worsens rapidly over a short period, often followed by periods of stability. Examples include sepsis, acute myocardial infarction (heart attack), and stroke.
3. Cyclical disease: In this type, the disease follows a cycle of worsening and improvement, with periodic exacerbations and remissions. Examples include multiple sclerosis, lupus, and rheumatoid arthritis.
4. Recurrent disease: This type is characterized by episodes of worsening followed by periods of recovery. Examples include migraine headaches, asthma, and appendicitis.
5. Catastrophic disease: In this type, the disease progresses rapidly and unpredictably, with a poor prognosis. Examples include cancer, AIDS, and organ failure.

Disease progression can be influenced by various factors, including:

1. Genetics: Some diseases are inherited and may have a predetermined course of progression.
2. Lifestyle: Factors such as smoking, lack of exercise, and poor diet can contribute to disease progression.
3. Environmental factors: Exposure to toxins, allergens, and other environmental stressors can influence disease progression.
4. Medical treatment: The effectiveness of medical treatment can impact disease progression, either by slowing or halting the disease process or by causing unintended side effects.
5. Co-morbidities: The presence of multiple diseases or conditions can interact and affect each other's progression.

Understanding the type and factors influencing disease progression is essential for developing effective treatment plans and improving patient outcomes.

There are several types of leukoencephalopathies, each with its own unique set of causes and characteristics. Some of the most common include:

1. Adrenoleukodystrophy (ALD): A genetic disorder that affects the breakdown of fatty acids in the body, leading to the accumulation of toxic substances in the brain.
2. Metachromatic leukodystrophy (MLD): A genetic disorder that affects the metabolism of certain fats in the body, leading to the accumulation of toxic substances in the brain.
3. Krabbe disease: A rare genetic disorder that affects the breakdown of a substance called galactocerebroside in the brain, leading to the accumulation of toxic substances and progressive damage to the nervous system.
4. Niemann-Pick disease: A group of inherited disorders that affect the metabolism of certain fats in the body, leading to the accumulation of toxic substances in the brain and other organs.
5. Alexander disease: A rare genetic disorder that affects the breakdown of a substance called galactose in the brain, leading to the accumulation of toxic substances and progressive damage to the nervous system.

The symptoms of leukoencephalopathies can vary depending on the specific type of disorder and the severity of the disease. Common symptoms include:

* Cognitive impairment: Difficulty with learning, memory, and problem-solving skills.
* Motor dysfunction: Weakness, rigidity, or tremors in the muscles.
* Seizures: Abnormal electrical activity in the brain that can cause convulsions or other symptoms.
* Vision loss: Blindness or vision impairment due to damage to the optic nerve.
* Speech difficulties: Slurred speech, difficulty with articulation, or other communication challenges.
* Behavioral changes: Increased irritability, aggression, or other behavioral problems.

There is no cure for leukoencephalopathies, but treatment options are available to manage the symptoms and slow the progression of the disease. These may include:

1. Physical therapy: To improve motor function and reduce muscle weakness.
2. Occupational therapy: To improve daily living skills and cognitive function.
3. Speech therapy: To improve communication skills and address swallowing difficulties.
4. Medications: To control seizures, muscle spasms, or other symptoms.
5. Nutritional support: To ensure adequate nutrition and address any feeding challenges.
6. Respiratory support: To assist with breathing and manage respiratory infections.
7. Psychological support: To address behavioral changes and other psychological issues.

The prognosis for leukoencephalopathies is generally poor, as the diseases tend to progress rapidly and can lead to significant disability or death within a few years of onset. However, with appropriate management and support, many individuals with these conditions can achieve a good quality of life and live well into adulthood. It is important for families to work closely with healthcare providers to develop a comprehensive treatment plan that addresses their child's specific needs and provides ongoing support throughout their lives.

Note: Hematoma is a collection of blood outside the blood vessels.

Some common types of movement disorders include:

1. Parkinson's disease: A degenerative disorder characterized by tremors, rigidity, bradykinesia, and postural instability.
2. Dystonia: A movement disorder characterized by sustained or intermittent muscle contractions that cause abnormal postures or movements.
3. Huntington's disease: An inherited disorder that causes progressive damage to the brain, leading to involuntary movements, cognitive decline, and psychiatric symptoms.
4. Tourette syndrome: A neurodevelopmental disorder characterized by repetitive, involuntary movements and vocalizations (tics).
5. Restless leg syndrome: A condition characterized by an uncomfortable sensation in the legs, often described as a creeping or crawling feeling, which is relieved by movement.
6. Chorea: A movement disorder characterized by rapid, jerky movements that can be triggered by emotional stress or other factors.
7. Ballism: Excessive, large, and often circular movements of the limbs, often seen in conditions such as Huntington's disease or drug-induced movements.
8. Athetosis: A slow, writhing movement that can be seen in conditions such as cerebral palsy or tardive dyskinesia.
9. Myoclonus: Sudden, brief muscle jerks or twitches that can be caused by a variety of factors, including genetic disorders, infections, and certain medications.
10. Hyperkinesis: An excessive amount of movement, often seen in conditions such as attention deficit hyperactivity disorder (ADHD) or hyperthyroidism.

Movement disorders can significantly impact an individual's quality of life, and treatment options vary depending on the specific condition and its underlying cause. Some movement disorders may be managed with medication, while others may require surgery or other interventions.

1. Infection: Bacterial or viral infections can develop after surgery, potentially leading to sepsis or organ failure.
2. Adhesions: Scar tissue can form during the healing process, which can cause bowel obstruction, chronic pain, or other complications.
3. Wound complications: Incisional hernias, wound dehiscence (separation of the wound edges), and wound infections can occur.
4. Respiratory problems: Pneumonia, respiratory failure, and atelectasis (collapsed lung) can develop after surgery, particularly in older adults or those with pre-existing respiratory conditions.
5. Cardiovascular complications: Myocardial infarction (heart attack), cardiac arrhythmias, and cardiac failure can occur after surgery, especially in high-risk patients.
6. Renal (kidney) problems: Acute kidney injury or chronic kidney disease can develop postoperatively, particularly in patients with pre-existing renal impairment.
7. Neurological complications: Stroke, seizures, and neuropraxia (nerve damage) can occur after surgery, especially in patients with pre-existing neurological conditions.
8. Pulmonary embolism: Blood clots can form in the legs or lungs after surgery, potentially causing pulmonary embolism.
9. Anesthesia-related complications: Respiratory and cardiac complications can occur during anesthesia, including respiratory and cardiac arrest.
10. delayed healing: Wound healing may be delayed or impaired after surgery, particularly in patients with pre-existing medical conditions.

It is important for patients to be aware of these potential complications and to discuss any concerns with their surgeon and healthcare team before undergoing surgery.

The term "infarction" is derived from the Latin words "in" meaning "into" and "farcire" meaning "to stuff", which refers to the idea that the tissue becomes "stuffed" with blood, leading to cell death and necrosis.

Infarction can be caused by a variety of factors, including atherosclerosis (the buildup of plaque in the blood vessels), embolism (a blood clot or other foreign material that blocks the flow of blood), and vasospasm (constriction of the blood vessels).

The symptoms of infarction vary depending on the location and severity of the blockage, but can include chest pain or discomfort, shortness of breath, numbness or weakness in the affected limbs, and confusion or difficulty speaking or understanding speech.

Diagnosis of infarction typically involves imaging tests such as electrocardiograms (ECGs), echocardiograms, or computerized tomography (CT) scans to confirm the presence of a blockage and assess the extent of the damage. Treatment options for infarction include medications to dissolve blood clots, surgery to restore blood flow, and other interventions to manage symptoms and prevent complications.

Prevention of infarction involves managing risk factors such as high blood pressure, high cholesterol, smoking, and obesity, as well as maintaining a healthy diet and exercise routine. Early detection and treatment of blockages can help reduce the risk of infarction and minimize the damage to affected tissues.

Types of foot deformities include:

1. Bunions: A bony growth on the side of the big toe that can cause pain and discomfort.
2. Hammertoes: A deformed toe caused by a muscle imbalance, which can lead to pain and corns.
3. Clubfoot: A condition in which the foot is turned inward or outward at birth.
4. Flat feet: A condition in which the arch of the foot collapses, causing the sole to be flat.
5. High arches: An abnormal curvature of the foot that can cause pain and stiffness.
6. Plantar fasciitis: Inflammation of the tissue on the bottom of the foot, which can cause heel pain.
7. Achilles tendinitis: Inflammation of the tendon that connects the calf muscle to the heel bone.
8. Bursitis: Inflammation of the fluid-filled sac (bursa) that cushions the joints, causing pain and swelling.
9. Tailor's bunion: A bony growth on the fifth toe that can cause pain and corns.
10. Sesamoiditis: Inflammation of the small bones called sesamoids, which are located under the first metatarsal bone.

Symptoms of foot deformities can include:

* Pain or discomfort in the foot or ankle
* Difficulty walking or standing
* Swelling or redness
* Limited mobility or stiffness
* Corns or calluses
* Inflammation or warmth in the affected area

Causes of foot deformities can include:

* Genetics
* Injury or trauma
* Disease or infection
* Poorly fitting shoes or footwear
* Muscle imbalance or weakness
* Nerve damage or neurological conditions

Treatment options for foot deformities can include:

* Rest and ice to reduce pain and inflammation
* Physical therapy exercises to strengthen the muscles and improve flexibility
* Orthotics or shoe inserts to support the foot or ankle
* Medications to relieve pain and reduce inflammation
* Surgery to correct the deformity or repair damaged tissues.

It is important to seek medical attention if you experience any persistent pain or discomfort in your feet, as early treatment can help prevent further damage and improve outcomes.

There are two types of heart arrest:

1. Asystole - This is when the heart stops functioning completely and there is no electrical activity in the heart.
2. Pulseless ventricular tachycardia or fibrillation - This is when the heart is still functioning but there is no pulse and the rhythm is abnormal.

Heart arrest can be diagnosed through various tests such as electrocardiogram (ECG), blood tests, and echocardiography. Treatment options for heart arrest include cardiopulmonary resuscitation (CPR), defibrillation, and medications to restore a normal heart rhythm.

In severe cases of heart arrest, the patient may require advanced life support measures such as mechanical ventilation and cardiac support devices. The prognosis for heart arrest is generally poor, especially if it is not treated promptly and effectively. However, with proper treatment and support, some patients can recover and regain normal heart function.

These disorders can cause a range of symptoms including cognitive impairment, confusion, memory loss, seizures, and changes in behavior and mood. Treatment options for brain disease metabolic disorders vary depending on the specific condition and may include medication, lifestyle changes, and other interventions such as surgery or rehabilitation therapy.

Examples of brain diseases, metabolic include:

* Hypoglycemia (low blood sugar)
* Hyperglycemia (high blood sugar)
* Diabetes mellitus (type 1 and type 2)
* Metabolic stroke
* Traumatic brain injury
* Neurodegenerative diseases such as Alzheimer's disease, Parkinson's disease, and Huntington's disease.

It is important to note that while these conditions are considered metabolic disorders, they can also have a significant impact on other aspects of an individual's life, including their mood, behavior, and cognitive functioning. Therefore, it is important to seek medical attention if symptoms persist or worsen over time.

Epilepsy, temporal lobe can cause a variety of seizure types, including:

1. Partial seizures: These are seizures that affect only one part of the brain, such as the temporal lobe.
2. Simple partial seizures: These are seizures that do not involve convulsions or loss of consciousness.
3. Complex partial seizures: These are seizures that involve impaired awareness or altered perception, and may involve convulsions or muscle stiffness.
4. Tonic-clonic seizures (formerly known as grand mal seizures): These are seizures that involve convulsions, loss of consciousness, and muscle stiffness.

The symptoms of epilepsy, temporal lobe can vary depending on the location of the seizure focus within the temporal lobe and the individual's age, but may include:

1. Auras (sensory disturbances such as flashing lights or unusual smells)
2. Confusion or disorientation
3. Memory loss or difficulty with memory
4. Emotional changes (such as fear, anxiety, or euphoria)
5. Speech difficulties
6. Muscle stiffness or weakness
7. Coordination problems
8. Vision changes (such as blurred vision or double vision)
9. Hearing changes (such as ringing in the ears)
10. Numbness or tingling sensations

Epilepsy, temporal lobe is typically diagnosed using a combination of medical history, physical examination, and diagnostic tests such as electroencephalography (EEG) or magnetic resonance imaging (MRI). Treatment options may include medication, surgery, or lifestyle modifications.

In some cases, hyperemia can be a sign of a more serious underlying condition that requires medical attention. For example, if hyperemia is caused by an inflammatory or infectious process, it may lead to tissue damage or organ dysfunction if left untreated.

Hyperemia can occur in various parts of the body, including the skin, muscles, organs, and other tissues. It is often diagnosed through physical examination and imaging tests such as ultrasound, computed tomography (CT), or magnetic resonance imaging (MRI). Treatment for hyperemia depends on its underlying cause, and may include antibiotics, anti-inflammatory medications, or surgery.

In the context of dermatology, hyperemia is often used to describe a condition called erythema, which is characterized by redness and swelling of the skin due to increased blood flow. Erythema can be caused by various factors, such as sun exposure, allergic reactions, or skin infections. Treatment for erythema may include topical medications, oral medications, or other therapies depending on its underlying cause.

Example: "The patient suffered a cerebral hemorrhage as a result of a car accident, which led to severe brain damage."

Fat embolism is a condition that occurs when fat droplets enter the bloodstream and get stuck in a blood vessel, causing a blockage. This can lead to serious complications, such as respiratory failure or death.

Causes of Fat Embolism
---------------------

Fat embolism can occur due to various reasons, including:

* Trauma or injury: A blow to the body can cause fat droplets to enter the bloodstream.
* Surgery: Certain surgeries, such as hip replacement or knee replacement, can disrupt the fat tissue and cause it to enter the bloodstream.
* Cancer: Some types of cancer, such as multiple myeloma or osteosarcoma, can cause fat embolism.
* Bone fractures: A fracture in a bone can cause fat droplets to enter the bloodstream.

Symptoms of Fat Embolism
-------------------------

The symptoms of fat embolism can vary depending on the severity of the condition. Some common symptoms include:

* Shortness of breath
* Chest pain or discomfort
* Rapid heart rate
* Coughing up blood
* Confusion or altered mental state
* Weakness or numbness in the legs
* Seizures

Diagnosis and Treatment of Fat Embolism
---------------------------------------

Diagnosing fat embolism can be challenging, as it may resemble other conditions such as pulmonary embolism or pneumonia. However, a doctor may use various tests, including:

* Chest X-ray: To look for signs of fluid accumulation in the lungs.
* CT scan: To visualize the fat droplets in the blood vessels.
* Blood tests: To check for signs of inflammation or infection.

Treatment for fat embolism typically involves supportive care, such as:

* Oxygen therapy: To help improve oxygen levels in the body.
* Pain management: To relieve chest pain and discomfort.
* Antibiotics: To prevent or treat any secondary infections.
* Medications to dissolve blood clots: To prevent further complications.

In severe cases, surgical intervention may be necessary to remove the fat droplets from the blood vessels.

Prevention of Fat Embolism
------------------------

Preventing fat embolism can be challenging, as it is a rare condition that can occur unexpectedly. However, there are some measures that may help reduce the risk, such as:

* Maintaining a healthy weight: To reduce the amount of fat that can enter the bloodstream.
* Exercising regularly: To improve circulation and reduce the risk of blood clots.
* Avoiding long periods of immobility: To reduce the risk of blood clots forming in the legs.

Conclusion
----------

Fat embolism is a rare but potentially life-threatening condition that can occur when fat enters the bloodstream and causes blockages in the blood vessels. Early diagnosis and treatment are crucial to prevent complications and improve outcomes. If you suspect you or someone else may have fat embolism, seek medical attention immediately.

There are different types of blindness, including:

1. Congenital blindness: Blindness that is present at birth, often due to genetic mutations or abnormalities in the development of the eye and brain.
2. Acquired blindness: Blindness that develops later in life due to injury, disease, or other factors.
3. Amblyopia: A condition where one eye has reduced vision due to misalignment or other causes.
4. Glaucoma: A group of eye conditions that can damage the optic nerve and lead to blindness if left untreated.
5. Retinitis pigmentosa: A degenerative disease that affects the retina and can cause blindness.
6. Cataracts: A clouding of the lens in the eye that can impair vision and eventually cause blindness if left untreated.
7. Macular degeneration: A condition where the macula, a part of the retina responsible for central vision, deteriorates and causes blindness.

There are various treatments and therapies for blindness, depending on the underlying cause. These may include medications, surgery, low vision aids, and assistive technology such as braille and audio books, screen readers, and voice-controlled software. Rehabilitation programs can also help individuals adapt to blindness and lead fulfilling lives.

Gliosis is made up of glial cells, which are non-neuronal cells that provide support and protection to neurons. When neural tissue is damaged, glial cells proliferate and form a scar-like tissue to fill in the gap and repair the damage. This scar tissue can be made up of astrocytes, oligodendrocytes, or microglia, depending on the type of injury and the location of the damage.

Gliosis can have both beneficial and harmful effects on the brain. On one hand, it can help to prevent further damage by providing a physical barrier against invading substances and protecting the surrounding neural tissue. It can also promote healing by bringing in immune cells and growth factors that aid in the repair process.

On the other hand, gliosis can also have negative effects on brain function. The scar tissue can disrupt normal communication between neurons, leading to impaired cognitive and motor function. In addition, if the scar tissue is too extensive or severe, it can compress or displaces surrounding neural tissue, leading to long-term neurological deficits or even death.

There are several ways to diagnose gliosis, including magnetic resonance imaging (MRI), positron emission tomography (PET), and histopathology. Treatment options for gliosis depend on the underlying cause of the condition and can include medications, surgery, or a combination of both.

In summary, gliosis is a type of scar tissue that forms in the brain and spinal cord as a result of damage to neural tissue. It can have both beneficial and harmful effects on brain function, and diagnosis and treatment options vary depending on the underlying cause of the condition.

The symptoms of altitude sickness can vary in severity and may include:

* Headache
* Dizziness and lightheadedness
* Nausea and vomiting
* Fatigue and weakness
* Shortness of breath
* Coughing and chest tightness
* Swelling of the hands, feet, and face

In severe cases, altitude sickness can lead to more serious complications such as:

* High-altitude pulmonary edema (HAPE): fluid buildup in the lungs that can be life-threatening
* High-altitude cerebral edema (HACE): fluid buildup in the brain that can be life-threatening

To prevent altitude sickness, it is recommended to ascend gradually and give your body time to acclimate to the higher altitude. This can be done by spending a few days at a lower altitude before ascending to a higher altitude. It is also important to stay hydrated by drinking plenty of water and avoid alcohol and sedatives, which can increase the risk of altitude sickness.

If you experience any symptoms of altitude sickness, it is important to descend to a lower altitude as soon as possible. Medications such as acetazolamide (Diamox) can also be used to help prevent and treat altitude sickness. In severe cases, hospitalization may be necessary to receive oxygen therapy and other medical treatment.

Dysarthria can affect both children and adults, and the symptoms can vary in severity depending on the underlying cause of the condition. Some common symptoms of dysarthria include:

* Slurred or slow speech
* Difficulty articulating words
* Poor enunciation
* Stuttering or hesitation while speaking
* Difficulty with word-finding and language processing
* Limited range of speech sounds
* Difficulty with loudness and volume control

Dysarthria can be diagnosed by a speech-language pathologist (SLP), who will typically conduct a comprehensive evaluation of the individual's speech and language abilities. This may include a series of tests to assess the individual's articulation, fluency, voice quality, and other aspects of their speech.

There are several types of dysarthria, including:

* Hypokinetic dysarthria: characterized by reduced muscle tone and slow movement of the articulatory organs, resulting in slurred or slow speech.
* Hyperkinetic dysarthria: characterized by increased muscle tone and rapid movement of the articulatory organs, resulting in fast but imprecise speech.
* Mixed dysarthria: a combination of hypokinetic and hyperkinetic features.
* Dystonic dysarthria: characterized by involuntary movements and postures of the tongue and lips, resulting in distorted speech.

Treatment for dysarthria typically involves speech therapy with an SLP, who will work with the individual to improve their speech clarity, fluency, and overall communication skills. Treatment may include exercises to strengthen the muscles used in speech production, as well as strategies to improve articulation, pronunciation, and language processing. In some cases, technology such as speech-generating devices may be used to support communication.

In addition to speech therapy, treatment for dysarthria may also involve other healthcare professionals, such as neurologists, physical therapists, or occupational therapists, depending on the underlying cause of the condition.

Overall, dysarthria is a speech disorder that can significantly impact an individual's ability to communicate effectively. However, with the right treatment and support from healthcare professionals and SLPs, many people with dysarthria are able to improve their communication skills and lead fulfilling lives.

Developmental disabilities can include a wide range of diagnoses, such as:

1. Autism Spectrum Disorder (ASD): A neurological disorder characterized by difficulties with social interaction, communication, and repetitive behaviors.
2. Intellectual Disability (ID): A condition in which an individual's cognitive abilities are below average, affecting their ability to learn, reason, and communicate.
3. Down Syndrome: A genetic disorder caused by an extra copy of chromosome 21, characterized by intellectual disability, delayed speech and language development, and a distinctive physical appearance.
4. Cerebral Palsy (CP): A group of disorders that affect movement, balance, and posture, often resulting from brain injury or abnormal development during fetal development or early childhood.
5. Attention Deficit Hyperactivity Disorder (ADHD): A neurodevelopmental disorder characterized by symptoms of inattention, hyperactivity, and impulsivity.
6. Learning Disabilities: Conditions that affect an individual's ability to learn and process information, such as dyslexia, dyscalculia, and dysgraphia.
7. Traumatic Brain Injury (TBI): An injury to the brain caused by a blow or jolt to the head, often resulting in cognitive, emotional, and physical impairments.
8. Severe Hearing or Vision Loss: A condition in which an individual experiences significant loss of hearing or vision, affecting their ability to communicate and interact with their environment.
9. Multiple Disabilities: A condition in which an individual experiences two or more developmental disabilities simultaneously, such as intellectual disability and autism spectrum disorder.
10. Undiagnosed Developmental Delay (UDD): A condition in which an individual experiences delays in one or more areas of development, but does not meet the diagnostic criteria for a specific developmental disability.

These conditions can have a profound impact on an individual's quality of life, and it is important to provide appropriate support and accommodations to help them reach their full potential.

1. Migraine: A severe, recurring headache that can last for hours to days, often accompanied by sensitivity to light, sound, and nausea.
2. Tension-type headache: A common type of headache characterized by a dull, aching pain on both sides of the head, often associated with muscle tension.
3. Cluster headache: A severe, one-sided headache that can occur multiple times a day and last for weeks or months.
4. Trigeminal autonomic cephalalgias (TACs): A group of rare but severely painful headaches that are often associated with facial swelling and tearing.

Primary headache disorders can be challenging to diagnose and manage, as they can have overlapping symptoms and may not be easily distinguishable from other conditions. However, there are several medications and therapies available to help manage these disorders and improve quality of life for individuals affected by them.

This can happen for various reasons, such as:

1. Prolonged labor or difficult delivery
2. Umbilical cord compression or knotting
3. Fetal distress or heart rate abnormalities during delivery
4. Maternal hypertension or pre-eclampsia
5. Placental abruption or placental insufficiency
6. Infection in the mother or baby during pregnancy or delivery
7. Drug or alcohol exposure during pregnancy
8. Maternal trauma or shock during delivery
9. Fetal growth restriction or small for gestational age
10. Congenital anomalies or birth defects

The symptoms of asphyxia neonatorum can vary depending on the severity and duration of the oxygen deprivation, but may include:

1. Cyanosis (blue skin color)
2. Apnea (pauses in breathing)
3. Bradycardia (slow heart rate)
4. Hypotonia (low muscle tone)
5. Poor reflexes
6. Seizures or convulsions
7. Gradual decline in muscle tone and organ function over time
8. Increased risk of infection or sepsis
9. Neurological damage, including cerebral palsy or cognitive impairment
10. Mortality (death)

Asphyxia neonatorum is a medical emergency that requires immediate attention and treatment. Treatment may include oxygen therapy, mechanical ventilation, and other supportive care to help the baby recover from the asphyxial event. In severe cases, asphyxia neonatorum can lead to long-term disabilities or death, so it is crucial to identify and treat the underlying causes promptly and effectively.

Some common examples of intraoperative complications include:

1. Bleeding: Excessive bleeding during surgery can lead to hypovolemia (low blood volume), anemia (low red blood cell count), and even death.
2. Infection: Surgical wounds can become infected, leading to sepsis or bacteremia (bacterial infection of the bloodstream).
3. Nerve damage: Surgery can sometimes result in nerve damage, leading to numbness, weakness, or paralysis.
4. Organ injury: Injury to organs such as the liver, lung, or bowel can occur during surgery, leading to complications such as bleeding, infection, or organ failure.
5. Anesthesia-related complications: Problems with anesthesia can include respiratory or cardiac depression, allergic reactions, or awareness during anesthesia (a rare but potentially devastating complication).
6. Hypotension: Low blood pressure during surgery can lead to inadequate perfusion of vital organs and tissues, resulting in organ damage or death.
7. Thromboembolism: Blood clots can form during surgery and travel to other parts of the body, causing complications such as stroke, pulmonary embolism, or deep vein thrombosis.
8. Postoperative respiratory failure: Respiratory complications can occur after surgery, leading to respiratory failure, pneumonia, or acute respiratory distress syndrome (ARDS).
9. Wound dehiscence: The incision site can separate or come open after surgery, leading to infection, fluid accumulation, or hernia.
10. Seroma: A collection of serous fluid that can develop at the surgical site, which can become infected and cause complications.
11. Nerve damage: Injury to nerves during surgery can result in numbness, weakness, or paralysis, sometimes permanently.
12. Urinary retention or incontinence: Surgery can damage the bladder or urinary sphincter, leading to urinary retention or incontinence.
13. Hematoma: A collection of blood that can develop at the surgical site, which can become infected and cause complications.
14. Pneumonia: Inflammation of the lungs after surgery can be caused by bacteria, viruses, or fungi and can lead to serious complications.
15. Sepsis: A systemic inflammatory response to infection that can occur after surgery, leading to organ dysfunction and death if not treated promptly.

It is important to note that these are potential complications, and not all patients will experience them. Additionally, many of these complications are rare, and the vast majority of surgeries are successful with minimal or no complications. However, it is important for patients to be aware of the potential risks before undergoing surgery so they can make an informed decision about their care.

Some examples of pathologic constrictions include:

1. Stenosis: A narrowing or constriction of a blood vessel or other tubular structure, often caused by the buildup of plaque or scar tissue.
2. Asthma: A condition characterized by inflammation and constriction of the airways, which can make breathing difficult.
3. Esophageal stricture: A narrowing of the esophagus that can cause difficulty swallowing.
4. Gastric ring constriction: A narrowing of the stomach caused by a band of tissue that forms in the upper part of the stomach.
5. Anal fissure: A tear in the lining of the anus that can cause pain and difficulty passing stools.

Pathologic constrictions can be caused by a variety of factors, including inflammation, infection, injury, or genetic disorders. They can be diagnosed through imaging tests such as X-rays, CT scans, or endoscopies, and may require surgical treatment to relieve symptoms and improve function.

There are several types of aphasia, including:

1. Broca's aphasia: Characterized by difficulty speaking in complete sentences and using correct grammar.
2. Wernicke's aphasia: Characterized by difficulty understanding spoken language and speaking in complete sentences.
3. Global aphasia: Characterized by a severe impairment of all language abilities.
4. Primary progressive aphasia: A rare form of aphasia that is caused by neurodegeneration and worsens over time.

Treatment for aphasia typically involves speech and language therapy, which can help individuals with aphasia improve their communication skills and regain some of their language abilities. Other forms of therapy, such as cognitive training and physical therapy, may also be helpful.

It's important to note that while aphasia can significantly impact an individual's quality of life, it does not affect their intelligence or cognitive abilities. With appropriate treatment and support, individuals with aphasia can continue to lead fulfilling lives and communicate effectively with others.

There are several possible causes of orthostatic hypotension, including:

1. Deconditioning: This is a common cause of orthostatic hypotension in older adults who have been bedridden or hospitalized for prolonged periods.
2. Medication side effects: Certain medications, such as beta blockers and vasodilators, can cause orthostatic hypotension as a side effect.
3. Heart conditions: Conditions such as heart failure, arrhythmias, and structural heart defects can lead to orthostatic hypotension.
4. Neurological disorders: Certain neurological disorders, such as Parkinson's disease, multiple sclerosis, and stroke, can cause orthostatic hypotension.
5. Vasomotor instability: This is a condition where the blood vessels constrict or dilate rapidly, leading to a drop in blood pressure.
6. Anemia: A low red blood cell count can lead to a decrease in oxygen delivery to the body's tissues, causing orthostatic hypotension.
7. Dehydration: Dehydration can cause a drop in blood volume and lead to orthostatic hypotension.
8. Hypovolemia: This is a condition where there is a low volume of blood in the body, leading to a drop in blood pressure.
9. Sepsis: Sepsis can cause vasodilation and lead to orthostatic hypotension.
10. Other causes: Other causes of orthostatic hypotension include adrenal insufficiency, thyroid disorders, and certain genetic conditions.

Symptoms of orthostatic hypotension may include:

* Dizziness or lightheadedness
* Fainting
* Blurred vision
* Nausea and vomiting
* Headaches
* Fatigue
* Weakness
* Confusion

If you experience any of these symptoms, it is important to seek medical attention as soon as possible. Your healthcare provider can perform a physical examination and order diagnostic tests to determine the underlying cause of your orthostatic hypotension. Treatment will depend on the specific cause, but may include medications to raise blood pressure, fluid replacement, and addressing any underlying conditions.

There are several types of dementia, each with its own set of symptoms and characteristics. Some common types of dementia include:

* Alzheimer's disease: This is the most common form of dementia, accounting for 50-70% of all cases. It is a progressive disease that causes the death of brain cells, leading to memory loss and cognitive decline.
* Vascular dementia: This type of dementia is caused by problems with blood flow to the brain, often as a result of a stroke or small vessel disease. It can cause difficulty with communication, language, and visual-spatial skills.
* Lewy body dementia: This type of dementia is characterized by the presence of abnormal protein deposits called Lewy bodies in the brain. It can cause a range of symptoms, including memory loss, confusion, hallucinations, and difficulty with movement.
* Frontotemporal dementia: This is a group of diseases that affect the front and temporal lobes of the brain, leading to changes in personality, behavior, and language.

The symptoms of dementia can vary depending on the underlying cause, but common symptoms include:

* Memory loss: Difficulty remembering recent events or learning new information.
* Communication and language difficulties: Struggling to find the right words or understand what others are saying.
* Disorientation: Getting lost in familiar places or having difficulty understanding the time and date.
* Difficulty with problem-solving: Trouble with planning, organizing, and decision-making.
* Mood changes: Depression, anxiety, agitation, or aggression.
* Personality changes: Becoming passive, suspicious, or withdrawn.
* Difficulty with movement: Trouble with coordination, balance, or using utensils.
* Hallucinations: Seeing or hearing things that are not there.
* Sleep disturbances: Having trouble falling asleep or staying asleep.

The symptoms of dementia can be subtle at first and may progress slowly over time. In the early stages, they may be barely noticeable, but as the disease progresses, they can become more pronounced and interfere with daily life. It is important to seek medical advice if you or a loved one is experiencing any of these symptoms, as early diagnosis and treatment can help improve outcomes.

Examples of syndromes include:

1. Down syndrome: A genetic disorder caused by an extra copy of chromosome 21 that affects intellectual and physical development.
2. Turner syndrome: A genetic disorder caused by a missing or partially deleted X chromosome that affects physical growth and development in females.
3. Marfan syndrome: A genetic disorder affecting the body's connective tissue, causing tall stature, long limbs, and cardiovascular problems.
4. Alzheimer's disease: A neurodegenerative disorder characterized by memory loss, confusion, and changes in personality and behavior.
5. Parkinson's disease: A neurological disorder characterized by tremors, rigidity, and difficulty with movement.
6. Klinefelter syndrome: A genetic disorder caused by an extra X chromosome in males, leading to infertility and other physical characteristics.
7. Williams syndrome: A rare genetic disorder caused by a deletion of genetic material on chromosome 7, characterized by cardiovascular problems, developmental delays, and a distinctive facial appearance.
8. Fragile X syndrome: The most common form of inherited intellectual disability, caused by an expansion of a specific gene on the X chromosome.
9. Prader-Willi syndrome: A genetic disorder caused by a defect in the hypothalamus, leading to problems with appetite regulation and obesity.
10. Sjogren's syndrome: An autoimmune disorder that affects the glands that produce tears and saliva, causing dry eyes and mouth.

Syndromes can be diagnosed through a combination of physical examination, medical history, laboratory tests, and imaging studies. Treatment for a syndrome depends on the underlying cause and the specific symptoms and signs presented by the patient.

This condition can be caused by a variety of factors, including genetics, injury, or surgery. Treatment options for Equinus Deformity include physical therapy, bracing, and in some cases, surgery. The goal of treatment is to improve the range of motion in the foot and ankle, allowing the individual to walk and stand more comfortably.

In conclusion, Equinus Deformity is a condition that affects the foot and ankle, causing difficulty walking or standing due to limited range of motion. Treatment options are available, including physical therapy and surgery, to improve the condition and allow for greater mobility.

A sudden and unexpected tearing or breaking open of a bodily structure, such as a blood vessel, muscle, or tendon, without any obvious external cause. This can occur due to various factors, including genetic predisposition, aging, or other underlying medical conditions.

Examples:

* Spontaneous rupture of the Achilles tendon
* Spontaneous coronary artery dissection (SCAD)
* Spontaneous pneumothorax (collapsed lung)

Symptoms and Signs:

* Sudden, severe pain
* Swelling and bruising in the affected area
* Difficulty moving or using the affected limb
* Palpitations or shortness of breath (in cardiac cases)

Diagnosis:

* Physical examination and medical history
* Imaging tests, such as X-rays, CT scans, or MRI scans, to confirm the rupture and assess the extent of damage
* Blood tests to check for underlying conditions that may have contributed to the rupture

Treatment:

* Rest, ice, compression, and elevation (RICE) to reduce pain and swelling
* Immobilization of the affected limb with a cast or brace
* Medications to manage pain and inflammation
* Surgery may be required in some cases to repair the damaged tissue or organ

Prognosis:

* The prognosis for spontaneous rupture depends on the location and severity of the rupture, as well as the underlying cause. In general, the sooner treatment is received, the better the outcome.

Complications:

* Infection
* Further damage to surrounding tissues or organs
* Chronic pain or limited mobility
* In some cases, long-term disability or death

Recurrence can also refer to the re-emergence of symptoms in a previously treated condition, such as a chronic pain condition that returns after a period of remission.

In medical research, recurrence is often studied to understand the underlying causes of disease progression and to develop new treatments and interventions to prevent or delay its return.

The burden of chronic diseases is significant, with over 70% of deaths worldwide attributed to them, according to the World Health Organization (WHO). In addition to the physical and emotional toll they take on individuals and their families, chronic diseases also pose a significant economic burden, accounting for a large proportion of healthcare expenditure.

In this article, we will explore the definition and impact of chronic diseases, as well as strategies for managing and living with them. We will also discuss the importance of early detection and prevention, as well as the role of healthcare providers in addressing the needs of individuals with chronic diseases.

What is a Chronic Disease?

A chronic disease is a condition that lasts for an extended period of time, often affecting daily life and activities. Unlike acute diseases, which have a specific beginning and end, chronic diseases are long-term and persistent. Examples of chronic diseases include:

1. Diabetes
2. Heart disease
3. Arthritis
4. Asthma
5. Cancer
6. Chronic obstructive pulmonary disease (COPD)
7. Chronic kidney disease (CKD)
8. Hypertension
9. Osteoporosis
10. Stroke

Impact of Chronic Diseases

The burden of chronic diseases is significant, with over 70% of deaths worldwide attributed to them, according to the WHO. In addition to the physical and emotional toll they take on individuals and their families, chronic diseases also pose a significant economic burden, accounting for a large proportion of healthcare expenditure.

Chronic diseases can also have a significant impact on an individual's quality of life, limiting their ability to participate in activities they enjoy and affecting their relationships with family and friends. Moreover, the financial burden of chronic diseases can lead to poverty and reduce economic productivity, thus having a broader societal impact.

Addressing Chronic Diseases

Given the significant burden of chronic diseases, it is essential that we address them effectively. This requires a multi-faceted approach that includes:

1. Lifestyle modifications: Encouraging healthy behaviors such as regular physical activity, a balanced diet, and smoking cessation can help prevent and manage chronic diseases.
2. Early detection and diagnosis: Identifying risk factors and detecting diseases early can help prevent or delay their progression.
3. Medication management: Effective medication management is crucial for controlling symptoms and slowing disease progression.
4. Multi-disciplinary care: Collaboration between healthcare providers, patients, and families is essential for managing chronic diseases.
5. Health promotion and disease prevention: Educating individuals about the risks of chronic diseases and promoting healthy behaviors can help prevent their onset.
6. Addressing social determinants of health: Social determinants such as poverty, education, and employment can have a significant impact on health outcomes. Addressing these factors is essential for reducing health disparities and improving overall health.
7. Investing in healthcare infrastructure: Investing in healthcare infrastructure, technology, and research is necessary to improve disease detection, diagnosis, and treatment.
8. Encouraging policy change: Policy changes can help create supportive environments for healthy behaviors and reduce the burden of chronic diseases.
9. Increasing public awareness: Raising public awareness about the risks and consequences of chronic diseases can help individuals make informed decisions about their health.
10. Providing support for caregivers: Chronic diseases can have a significant impact on family members and caregivers, so providing them with support is essential for improving overall health outcomes.

Conclusion

Chronic diseases are a major public health burden that affect millions of people worldwide. Addressing these diseases requires a multi-faceted approach that includes lifestyle changes, addressing social determinants of health, investing in healthcare infrastructure, encouraging policy change, increasing public awareness, and providing support for caregivers. By taking a comprehensive approach to chronic disease prevention and management, we can improve the health and well-being of individuals and communities worldwide.

Symptoms of DKA can include:

* High blood sugar levels (usually above 300 mg/dL)
* High levels of ketones in the blood and urine
* Nausea, vomiting, and abdominal pain
* Fatigue, weakness, and confusion
* Headache and dry mouth
* Flu-like symptoms, such as fever, chills, and muscle aches

If left untreated, DKA can lead to serious complications, such as:

* Dehydration and electrolyte imbalances
* Seizures and coma
* Kidney damage and failure

Treatment of DKA typically involves hospitalization and intravenous fluids to correct dehydration and electrolyte imbalances. Insulin therapy is also started to lower blood sugar levels and promote the breakdown of ketones. In severe cases, medications such as sodium bicarbonate may be given to help neutralize the excess ketones in the blood.

Preventing DKA involves proper management of diabetes, including:

* Taking insulin as prescribed and monitoring blood sugar levels regularly
* Maintaining a healthy diet and exercise program
* Monitoring for signs of infection or illness, which can increase the risk of DKA

Early detection and treatment of DKA are critical to preventing serious complications and improving outcomes for people with diabetes.

There are many different types of nerve degeneration that can occur in various parts of the body, including:

1. Alzheimer's disease: A progressive neurological disorder that affects memory and cognitive function, leading to degeneration of brain cells.
2. Parkinson's disease: A neurodegenerative disorder that affects movement and balance, caused by the loss of dopamine-producing neurons in the brain.
3. Amyotrophic lateral sclerosis (ALS): A progressive neurological disease that affects nerve cells in the brain and spinal cord, leading to muscle weakness, paralysis, and eventually death.
4. Multiple sclerosis: An autoimmune disease that affects the central nervous system, causing inflammation and damage to nerve fibers.
5. Diabetic neuropathy: A complication of diabetes that can cause damage to nerves in the hands and feet, leading to pain, numbness, and weakness.
6. Guillain-Barré syndrome: An autoimmune disorder that can cause inflammation and damage to nerve fibers, leading to muscle weakness and paralysis.
7. Chronic inflammatory demyelinating polyneuropathy (CIDP): An autoimmune disorder that can cause inflammation and damage to nerve fibers, leading to muscle weakness and numbness.

The causes of nerve degeneration are not always known or fully understood, but some possible causes include:

1. Genetics: Some types of nerve degeneration may be inherited from one's parents.
2. Aging: As we age, our nerve cells can become damaged or degenerate, leading to a decline in cognitive and physical function.
3. Injury or trauma: Physical injury or trauma to the nervous system can cause nerve damage and degeneration.
4. Infections: Certain infections, such as viral or bacterial infections, can cause nerve damage and degeneration.
5. Autoimmune disorders: Conditions such as Guillain-Barré syndrome and chronic inflammatory demyelinating polyneuropathy (CIDP) are caused by the immune system attacking and damaging nerve cells.
6. Toxins: Exposure to certain toxins, such as heavy metals or pesticides, can damage and degenerate nerve cells.
7. Poor nutrition: A diet that is deficient in essential nutrients, such as vitamin B12 or other B vitamins, can lead to nerve damage and degeneration.
8. Alcoholism: Long-term alcohol abuse can cause nerve damage and degeneration due to the toxic effects of alcohol on nerve cells.
9. Drug use: Certain drugs, such as chemotherapy drugs and antiviral medications, can damage and degenerate nerve cells.
10. Aging: As we age, our nerve cells can deteriorate and become less functional, leading to a range of cognitive and motor symptoms.

It's important to note that in some cases, nerve damage and degeneration may be irreversible, but there are often strategies that can help manage symptoms and improve quality of life. If you suspect you have nerve damage or degeneration, it's important to seek medical attention as soon as possible to receive an accurate diagnosis and appropriate treatment.

Leukoaraiosis can cause a range of cognitive, motor, and behavioral symptoms depending on the severity and location of the degeneration. In some cases, it may lead to difficulty with walking, balance, and coordination, as well as cognitive impairments such as memory loss and difficulty with language processing.

The term "leukoaraiosis" comes from the Greek words "leukos," meaning white, and "araia," meaning matter. It is often used interchangeably with the term "white matter degeneration."

Leukoaraiosis can be diagnosed using a variety of neuroimaging techniques such as magnetic resonance imaging (MRI), diffusion tensor imaging (DTI), and magnetization transfer imaging (MTI). Treatment for leukoaraiosis is typically focused on managing the underlying cause of the condition, such as addressing inflammation or oxidative stress, and may include medications, physical therapy, and lifestyle changes.

In summary, leukoaraiosis refers to the loss or degeneration of white matter in the brain, leading to a range of cognitive, motor, and behavioral symptoms. It is often seen in neurodegenerative diseases and can be diagnosed using various neuroimaging techniques. Treatment is focused on managing the underlying cause of the condition.

Intracranial hematoma occurs within the skull and is often caused by head injuries, such as falls or car accidents. It can lead to severe neurological symptoms, including confusion, seizures, and loss of consciousness. Extracranial hematomas occur outside the skull and are commonly seen in injuries from sports, accidents, or surgery.

The signs and symptoms of hematoma may vary depending on its location and size. Common symptoms include pain, swelling, bruising, and limited mobility. Diagnosis is typically made through imaging tests such as CT scans or MRI scans, along with physical examination and medical history.

Treatment for hematoma depends on its severity and location. In some cases, conservative management with rest, ice, compression, and elevation (RICE) may be sufficient. However, surgical intervention may be necessary to drain the collection of blood or remove any clots that have formed.

In severe cases, hematoma can lead to life-threatening complications such as infection, neurological damage, and organ failure. Therefore, prompt medical attention is crucial for proper diagnosis and treatment.

Thalamic diseases can result from various causes, including genetic mutations, infections, trauma, and stroke. Some common thalamic diseases include:

1. Thalamic stroke or infarction: This occurs when there is a lack of blood supply to the thalamus, leading to cell death and loss of thalamic function.
2. Thalamic tumors: These are abnormal growths that can develop in the thalamus, either benign or malignant.
3. Thalamic lesions: These are areas of damage or degeneration in the thalamus, which can result from trauma, stroke, or other conditions such as multiple sclerosis.
4. Thalamic migraine: This is a type of migraine that is associated with activation of the thalamus and can cause severe headaches, visual disturbances, and other symptoms.
5. Thalamic pain disorders: These are conditions characterized by chronic pain that is thought to be related to dysfunction in the thalamus.
6. Thalamic sleep disorders: These are conditions that affect the regulation of sleep and wakefulness, such as narcolepsy or insomnia.
7. Thalamic cognitive disorders: These are conditions that affect cognitive function, such as memory loss, attention deficits, and language difficulties.

Thalamic diseases can be challenging to diagnose and treat, as the thalamus is a complex structure that is involved in many brain functions. However, advances in neuroimaging and other diagnostic tools have improved our ability to identify and understand these conditions. Treatment options for thalamic diseases vary depending on the specific condition and can range from medications and lifestyle changes to surgery and other interventions.

The symptoms of CNS helminthiasis can vary depending on the type of worm present, but may include:

* Seizures
* Headaches
* Vision problems
* Weakness or paralysis
* Confusion or disorientation
* Personality changes

CNS helminthiasis is caused by a variety of parasites, including tapeworms, roundworms, and hookworms. These worms can enter the body through contaminated food or water, or through contact with an infected person or animal.

The diagnosis of CNS helminthiasis typically involves a combination of physical examination, medical history, and laboratory tests such as blood tests or imaging studies (e.g., CT or MRI scans). Treatment usually involves anti-parasitic medications to kill the worms, and may also include supportive care to manage symptoms.

Preventive measures to avoid CNS helminthiasis include:

* Avoiding undercooked meat, especially pork and wild game
* Avoiding raw or undercooked fish and shellfish
* Avoiding contact with animals that may be infected
* Properly storing and cooking food to kill parasites
* Avoiding drinking untreated water
* Washing hands and fruits/vegetables before eating

Overall, CNS helminthiasis is a serious condition that can cause significant neurological symptoms. Prompt diagnosis and treatment are essential to prevent long-term damage and improve outcomes.

Astigmatism can occur in people of all ages and is usually present at birth, but it may not become noticeable until later in life. It may also develop as a result of an injury or surgery. Astigmatism can be corrected with glasses, contact lenses, or refractive surgery, such as LASIK.

There are different types of astigmatism, including:

1. Corneal astigmatism: This is the most common type of astigmatism and occurs when the cornea is irregularly shaped.
2. Lens astigmatism: This type of astigmatism occurs when the lens inside the eye is irregularly shaped.
3. Mixed astigmatism: This type of astigmatism occurs when both the cornea and lens are irregularly shaped.

Astigmatism can cause a range of symptoms, including:

* Blurred vision at all distances
* Distorted vision (such as seeing objects as being stretched out or blurry)
* Eye strain or fatigue
* Headaches or eye discomfort
* Squinting or tilting the head to see clearly

If you suspect you have astigmatism, it's important to see an eye doctor for a comprehensive eye exam. Astigmatism can be diagnosed with a visual acuity test and a retinoscopy, which measures the way the light enters the eye.

Astigmatism is a common vision condition that can be easily corrected with glasses, contact lenses, or refractive surgery. If you have astigmatism, it's important to seek professional treatment to improve your vision and reduce any discomfort or strain on the eyes.

Characteristics: People with cortical blindness may have difficulty recognizing objects, navigating their environment, and perceiving light and colors. They may also experience visual hallucinations or distortions. The blindness can be partial or total, and the degree of vision loss can vary widely.

Causes: Cortical blindness can be caused by a variety of factors, including:

* Stroke or brain injury that damages the visual cortex
* Infections such as meningitis or encephalitis that affect the visual cortex
* Genetic disorders such as retinitis pigmentosa or Leber's congenital amaurosis
* Traumatic brain injury
* Tumors or cysts in the visual cortex

Symptoms: Symptoms of cortical blindness can include:

* Difficulty recognizing objects or faces
* Poor spatial awareness and navigation
* Blurred or distorted vision
* Sensitivity to light or glare
* Visual hallucinations or distortions
* Partial or total loss of vision

Diagnosis: Diagnosis of cortical blindness typically involves a comprehensive eye exam, neurological evaluation, and imaging tests such as MRI or CT scans to identify any underlying causes.

Treatment: There is no cure for cortical blindness, but various therapies and strategies can help improve functional vision and independence. These may include:

* Vision rehabilitation therapy to improve visual function and adapt to new ways of seeing
* Assistive technology such as telescopes or closed-circuit television systems to enhance remaining vision
* Training in mobility and orientation skills
* Compensatory strategies for daily activities

Prognosis: The prognosis for cortical blindness varies depending on the underlying cause and severity of the condition. In some cases, partial recovery of vision may be possible with time and therapy, while in other instances, visual function may remain impaired or stable.

Prevention: Prevention of cortical blindness is not always possible, but early detection and treatment of underlying conditions can help reduce the risk of vision loss. Regular eye exams and monitoring by a neurologist or ophthalmologist can also help identify any changes in vision and potential causes of cortical blindness.

Overall, cortical blindness is a complex and rare condition that requires specialized care and rehabilitation to improve functional vision and independence. With the right therapies and strategies, individuals with cortical blindness can lead fulfilling lives and adapt to their new visual reality.

Articulation disorders can be classified into different types based on the severity and nature of the speech difficulties. Some common types of articulation disorders include:

1. Articulation errors: These occur when individuals produce speech sounds differently than the expected norm, such as pronouncing "k" and "s" sounds as "t" or "z."
2. Speech sound distortions: This type of disorder involves the exaggeration or alteration of speech sounds, such as speaking with a lisp or a nasal tone.
3. Speech articulation anomalies: These are abnormalities in the production of speech sounds that do not fit into any specific category, such as difficulty pronouncing certain words or sounds.
4. Apraxia of speech: This is a neurological disorder that affects the ability to plan and execute voluntary movements of the articulators (lips, tongue, jaw), resulting in distorted or slurred speech.
5. Dysarthria: This is a speech disorder characterized by weakness, slowness, or incoordination of the muscles used for speaking, often caused by a neurological condition such as a stroke or cerebral palsy.

Articulation disorders can be diagnosed by a speech-language pathologist (SLP) through a comprehensive evaluation of an individual's speech and language skills. The SLP may use standardized assessments, clinical observations, and interviews with the individual and their family to determine the nature and severity of the articulation disorder.

Treatment for articulation disorders typically involves speech therapy with an SLP, who will work with the individual to improve their speech skills through a series of exercises and activities tailored to their specific needs. Treatment may focus on improving the accuracy and clarity of speech sounds, increasing speech rate and fluency, and enhancing communication skills.

In addition to speech therapy, other interventions that may be helpful for individuals with articulation disorders include:

1. Augmentative and alternative communication (AAC) systems: For individuals with severe articulation disorders or those who have difficulty using speech to communicate, AAC systems such as picture communication symbols or electronic devices can provide an alternative means of communication.
2. Supportive technology: Assistive devices such as speech-generating devices, text-to-speech software, and other technology can help individuals with articulation disorders to communicate more effectively.
3. Parent-child interaction therapy (PCIT): This type of therapy focuses on improving the communication skills of young children with articulation disorders by training parents to use play-based activities and strategies to enhance their child's speech and language development.
4. Social skills training: For individuals with articulation disorders who also have difficulty with social interactions, social skills training can help them develop better communication and social skills.
5. Cognitive communication therapy: This type of therapy focuses on improving the cognitive processes that underlie communication, such as attention, memory, and problem-solving skills.
6. Articulation therapy: This type of therapy focuses specifically on improving articulation skills, and may involve exercises and activities to strengthen the muscles used for speech production.
7. Stuttering modification therapy: For individuals who stutter, this type of therapy can help them learn to speak more fluently and with less effort.
8. Voice therapy: This type of therapy can help individuals with voice disorders to improve their vocal quality and communication skills.
9. Counseling and psychotherapy: For individuals with articulation disorders who are experiencing emotional or psychological distress, counseling and psychotherapy can be helpful in addressing these issues and improving overall well-being.

It's important to note that the most effective treatment approach will depend on the specific needs and goals of the individual with an articulation disorder, as well as their age, severity of symptoms, and other factors. A speech-language pathologist can work with the individual and their family to develop a personalized treatment plan that addresses their unique needs and helps them achieve their communication goals.

Some common types of cerebellar diseases include:

1. Cerebellar atrophy: This is a condition where the cerebellum shrinks or degenerates, leading to symptoms such as tremors, muscle weakness, and difficulty with movement.
2. Cerebellar degeneration: This is a condition where the cerebellum deteriorates over time, leading to symptoms such as loss of coordination, balance problems, and difficulties with speech and language.
3. Cerebellar tumors: These are abnormal growths that develop in the cerebellum, which can cause a variety of symptoms depending on their size and location.
4. Cerebellar stroke: This is a condition where blood flow to the cerebellum is interrupted, leading to damage to the brain tissue and symptoms such as weakness or paralysis of certain muscle groups.
5. Cerebellar vasculature disorders: These are conditions that affect the blood vessels in the cerebellum, leading to symptoms such as transient ischemic attacks (TIAs) or strokes.
6. Inflammatory diseases: These are conditions that cause inflammation in the cerebellum, leading to symptoms such as tremors, ataxia, and weakness.
7. Infections: Bacterial, viral, or fungal infections can affect the cerebellum and cause a range of symptoms.
8. Trauma: Head injuries or other forms of trauma can damage the cerebellum and lead to symptoms such as loss of coordination, balance problems, and memory loss.
9. Genetic disorders: Certain genetic mutations can affect the development and function of the cerebellum, leading to a range of symptoms.
10. Degenerative diseases: Conditions such as multiple sclerosis, Parkinson's disease, and Huntington's disease can cause degeneration of the cerebellum and lead to symptoms such as tremors, ataxia, and weakness.

It's important to note that this is not an exhaustive list, and there may be other causes of cerebellar symptoms not included here. A healthcare professional can help determine the underlying cause of your symptoms based on a thorough medical history and examination.

The term "amyloid" refers specifically to the type of protein aggregate that forms these plaques, and is derived from the Greek word for "flour-like." Amyloidosis is the general term used to describe the condition of having amyloid deposits in the body, while Alzheimer's disease is a specific type of amyloidosis that is characterized by the accumulation of beta-amyloid peptides in the brain.

Plaques, amyloid play a central role in the pathogenesis of many neurodegenerative diseases, and understanding their formation and clearance is an area of ongoing research. In addition to their role in Alzheimer's disease, amyloid plaques have been implicated in other conditions such as cerebral amyloid angiopathy, primary lateral sclerosis, and progressive supranuclear palsy.

Plaques, amyloid are composed of a variety of proteins, including beta-amyloid peptides, tau protein, and apolipoprotein E (apoE). The composition and structure of these plaques can vary depending on the underlying disease, and their presence is often associated with inflammation and oxidative stress.

In addition to their role in neurodegeneration, amyloid plaques have been implicated in other diseases such as type 2 diabetes and cardiovascular disease. The accumulation of amyloid fibrils in these tissues can contribute to the development of insulin resistance and atherosclerosis, respectively.

Overall, plaques, amyloid are a complex and multifaceted area of research, with many open questions remaining about their formation, function, and clinical implications. Ongoing studies in this field may provide valuable insights into the pathogenesis of various diseases and ultimately lead to the development of novel therapeutic strategies for these conditions.

In conclusion, plaques, amyloid are a hallmark of several neurodegenerative diseases, including Alzheimer's disease, and have been associated with inflammation, oxidative stress, and neurodegeneration. The composition and structure of these plaques can vary depending on the underlying disease, and their presence is often linked to the progression of the condition. Furthermore, amyloid plaques have been implicated in other diseases such as type 2 diabetes and cardiovascular disease, highlighting their potential clinical significance beyond neurodegeneration. Ongoing research into the mechanisms of amyloid plaque formation and clearance may lead to the development of novel therapeutic strategies for these conditions.

Tonic movement:

* Stiffening or rigidity of muscles
* Loss of postural control

Clonic movement:

* Jerky movements of the arms, legs, or entire body
* Involuntary contractions

During a tonic-clonic seizure, the person may experience a variety of symptoms, including:

* Sudden loss of consciousness
* Confusion and disorientation after regaining consciousness
* Memory loss for the event
* Weakness or fatigue
* Headache
* Nausea and vomiting

Tonic-clonic seizures can be caused by a variety of factors, including:

* Genetic mutations that affect brain function
* Infections such as meningitis or encephalitis
* Traumatic head injury
* Stroke or bleeding in the brain
* Brain tumors or cysts
* Drug and alcohol withdrawal
* Electrolyte imbalances

There are several different types of tonic-clonic seizures, including:

* Simple partial seizures: These are less severe than tonic-clonic seizures and may involve only one part of the body.
* Complex partial seizures: These are more severe than simple partial seizures and can involve both sides of the body.
* Tonic-clonic seizures with secondary generalization: This type of seizure starts as a simple or complex partial seizure and then spreads to other parts of the body.

Treatment for tonic-clonic seizures typically involves medication, such as anticonvulsants, which can help reduce the frequency and severity of seizures. In some cases, surgery may be necessary to remove a brain tumor or cyst that is causing the seizures.

Overall, tonic-clonic seizures are a serious medical condition that can have significant consequences if not properly treated. If you experience a seizure, it is important to seek medical attention as soon as possible to determine the cause and receive appropriate treatment.

Examples of closed head injuries include:

* Concussions
* Contusions
* Cerebral edema (swelling of the brain)
* Brain hemorrhages (bleeding in the brain)

Closed head injuries can be caused by a variety of mechanisms, such as falls, motor vehicle accidents, sports injuries, and assaults.

Symptoms of closed head injuries may include:

* Headache
* Dizziness or loss of balance
* Confusion or disorientation
* Memory loss or difficulty concentrating
* Sleep disturbances
* Mood changes, such as irritability or depression
* Vision problems, such as blurred vision or sensitivity to light

Closed head injuries can be difficult to diagnose, as there may be no visible signs of injury. However, a healthcare provider may use imaging tests such as CT scans or MRI to look for evidence of damage to the brain. Treatment for closed head injuries typically involves rest, medication, and rehabilitation to help the patient recover from any cognitive, emotional, or physical symptoms. In some cases, surgery may be necessary to relieve pressure on the brain or repair damaged blood vessels.

The disorder is named after Dr. Schilder, a Dutch neurologist who first described it in 1928. It is also known as diffuse cerebral sclerosis, progressive cerebral degeneration, or Schilder's disease. The exact prevalence and incidence of the disorder are not known, but it is believed to affect approximately 1 in 1 million individuals worldwide.

The symptoms of Diffuse Cerebral Sclerosis of Schilder typically begin in early adulthood and progress slowly over several years. Affected individuals may experience cognitive decline, including memory loss, difficulty with concentration and problem-solving, and decreased language skills. They may also experience ataxia, which is a loss of coordination and balance, leading to difficulties with walking and maintaining their posture. Seizures are common in individuals with Diffuse Cerebral Sclerosis of Schilder, and can range from mild to severe. Weakness and paralysis may also develop as the disorder progresses.

The exact cause of Diffuse Cerebral Sclerosis of Schilder is not known, but it is believed to be related to an autoimmune response, in which the immune system mistakenly attacks healthy cells in the central nervous system. There is no cure for the disorder, and treatment is focused on managing symptoms and slowing its progression. Medications such as anticonvulsants and steroids may be used to control seizures and inflammation, while physical therapy and occupational therapy can help individuals maintain their physical function and independence.

In summary, Diffuse Cerebral Sclerosis of Schilder is a rare and progressive neurodegenerative disorder that affects the brain and spinal cord, leading to a range of cognitive, motor, and behavioral symptoms. While there is no cure for the disorder, treatment can help manage symptoms and slow its progression, allowing individuals with Diffuse Cerebral Sclerosis of Schilder to maintain their quality of life as much as possible.

There are several types of embolism, including:

1. Pulmonary embolism: A blood clot that forms in the lungs and blocks the flow of blood to the heart.
2. Cerebral embolism: A blood clot or other foreign substance that blocks the flow of blood to the brain.
3. Coronary embolism: A blood clot that blocks the flow of blood to the heart muscle, causing a heart attack.
4. Intestinal embolism: A blood clot or other foreign substance that blocks the flow of blood to the intestines.
5. Fat embolism: A condition where fat enters the bloodstream and becomes lodged in a blood vessel, blocking the flow of blood.

The symptoms of embolism can vary depending on the location of the blockage, but may include:

* Pain or tenderness in the affected area
* Swelling or redness in the affected limb
* Difficulty breathing or shortness of breath
* Chest pain or pressure
* Lightheadedness or fainting
* Rapid heart rate or palpitations

Treatment for embolism depends on the underlying cause and the severity of the blockage. In some cases, medication may be used to dissolve blood clots or break up the blockage. In other cases, surgery may be necessary to remove the foreign substance or repair the affected blood vessel.

Prevention is key in avoiding embolism, and this can include:

* Managing underlying conditions such as high blood pressure, diabetes, or heart disease
* Avoiding long periods of immobility, such as during long-distance travel
* Taking blood-thinning medication to prevent blood clots from forming
* Maintaining a healthy weight and diet to reduce the risk of fat embolism.

There are several types of acidosis, including:

1. Respiratory acidosis: This occurs when the lung's ability to remove carbon dioxide from the blood is impaired, leading to an increase in blood acidity.
2. Metabolic acidosis: This type of acidosis occurs when there is an excessive production of acid in the body due to factors such as diabetes, starvation, or kidney disease.
3. Mixed acidosis: This type of acidosis is a combination of respiratory and metabolic acidosis.
4. Severe acute respiratory acidosis (SARA): This is a life-threatening condition that occurs suddenly, usually due to a severe lung injury or aspiration of a corrosive substance.

The symptoms of acidosis can vary depending on the type and severity of the condition. Common symptoms include:

1. Fatigue
2. Weakness
3. Confusion
4. Headaches
5. Nausea and vomiting
6. Abdominal pain
7. Difficulty breathing
8. Rapid heart rate
9. Muscle twitching

If left untreated, acidosis can lead to complications such as:

1. Kidney damage
2. Seizures
3. Coma
4. Heart arrhythmias
5. Respiratory failure

Treatment of acidosis depends on the underlying cause and the severity of the condition. Some common treatments include:

1. Oxygen therapy
2. Medications to help regulate breathing and heart rate
3. Fluid and electrolyte replacement
4. Dietary changes
5. Surgery, in severe cases.

In conclusion, acidosis is a serious medical condition that can have severe consequences if left untreated. It is important to seek medical attention immediately if you suspect that you or someone else may have acidosis. With prompt and appropriate treatment, it is possible to effectively manage the condition and prevent complications.

Some common neurological manifestations include:

1. Weakness or paralysis of specific muscle groups
2. Numbness or tingling sensations in the limbs or body
3. Difficulty with speech, language, or swallowing
4. Vision problems, such as blurred vision, double vision, or loss of vision
5. Dizziness, vertigo, or loss of balance
6. Confusion, disorientation, or difficulty with memory
7. Seizures or convulsions
8. Headaches or migraines
9. Sleep disturbances, such as insomnia or narcolepsy
10. Behavioral changes, such as mood swings, depression, or anxiety

The neurological manifestations of a condition can vary depending on the underlying cause and the specific location and extent of the damage to the nervous system. Diagnosis and treatment of these manifestations require expertise in neurology and may involve a range of diagnostic tests, such as imaging studies, electromyography, and laboratory tests, as well as medications, surgery, or other interventions.

Esotropia is often diagnosed in children, and it can affect one or both eyes. Treatment for esotropia usually involves glasses or contact lenses to correct vision problems, as well as exercises to strengthen the muscles that control eye movement. In some cases, surgery may be necessary to realign the eyes.

Esotropia can also be associated with other conditions, such as craniosynostosis (a condition where the bones of the skull fuse together too early), or Down syndrome. It is important for parents and caregivers to be aware of the signs of esotropia, such as crossing or turning of the eyes, and to seek medical attention if they suspect that their child may have this condition. Early diagnosis and treatment can help prevent long-term vision problems and improve the overall quality of life for children with esotropia.

This condition is most commonly seen in people with advanced liver disease, such as cirrhosis or liver cancer. It can also be caused by other conditions that affect the liver, such as hepatitis or portal hypertension.

Symptoms of hepatic encephalopathy can include confusion, disorientation, slurred speech, memory loss, and difficulty with coordination and balance. In severe cases, it can lead to coma or even death.

Diagnosis of hepatic encephalopathy is typically made through a combination of physical examination, medical history, and diagnostic tests such as blood tests and imaging studies. Treatment options include medications to reduce the production of ammonia in the gut, antibiotics to treat any underlying infections, and transjugular intrahepatic portosystemic shunt (TIPS) to improve liver function. In severe cases, a liver transplant may be necessary.

Overall, hepatic encephalopathy is a serious condition that can have significant impact on quality of life and survival in people with advanced liver disease. Early detection and prompt treatment are essential to prevent complications and improve outcomes.

Symptoms of hydrocephalus, normal pressure can include headaches, nausea and vomiting, double vision, and difficulty with balance and coordination. However, unlike hydrocephalus, elevated pressure, which is caused by an excessive accumulation of CSF, the symptoms of hydrocephalus, normal pressure are usually milder and may not be as severe.

Treatment options for hydrocephalus, normal pressure can include medications to relieve symptoms, such as headaches and nausea, as well as surgery to drain excess CSF or to repair any blockages or abnormalities in the flow of CSF. In some cases, a shunt may be inserted to drain excess CSF into another part of the body, such as the abdomen.

The symptoms of neuroaspergillosis can vary depending on the location and severity of the infection, but may include:

* Headache
* Fever
* Confusion
* Seizures
* Weakness or paralysis
* Vision problems
* Hearing loss

In severe cases, neuroaspergillosis can lead to brain abscesses, stroke, or death.

The diagnosis of neuroaspergillosis is based on a combination of clinical findings, laboratory tests, and imaging studies such as CT or MRI scans. Laboratory tests may include blood cultures, cerebrospinal fluid (CSF) analysis, and PCR testing for Aspergillus DNA.

Treatment of neuroaspergillosis typically involves a combination of antifungal medications and supportive care to manage symptoms and prevent complications. In some cases, surgical intervention may be necessary to drain abscesses or relieve pressure on the brain.

Prevention of neuroaspergillosis primarily involves managing underlying conditions that weaken the immune system, such as HIV/AIDS or cancer, and avoiding exposure to Aspergillus fungi. In high-risk individuals, prophylactic antifungal medications may be used to prevent infection.

Other definitions:

* Premature birth: A birth that occurs before 37 completed weeks of gestation.
* Preterm birth: A birth that occurs before 37 completed weeks of gestation, but not necessarily before 22 weeks.
* Very preterm birth: A birth that occurs before 28 completed weeks of gestation.
* Extremely preterm birth: A birth that occurs before 24 completed weeks of gestation.

Diseases associated with premature infants:

1. Respiratory distress syndrome (RDS): A condition in which the baby's lungs do not produce enough surfactant, a substance that helps the air sacs in the lungs expand and contract properly.
2. Bronchopulmonary dysplasia (BPD): A chronic lung disease that can develop in premature infants who have RDS.
3. Intraventricular hemorrhage (IVH): Bleeding in the brain that can occur in premature infants, particularly those with RDS or BPD.
4. Retinopathy of prematurity (ROP): A condition that can cause blindness in premature infants due to abnormal blood vessel growth in the retina.
5. Necrotizing enterocolitis (NEC): A condition that can cause damage to the intestines and other parts of the digestive system in premature infants.
6. Intracranial hemorrhage (ICH): Bleeding in the brain that can occur in premature infants, particularly those with RDS or BPD.
7. Gastrointestinal problems: Premature infants are at risk for gastroesophageal reflux disease (GERD), necrotizing enterocolitis (NEC), and other gastrointestinal problems.
8. Feeding difficulties: Premature infants may have difficulty feeding, which can lead to weight gain issues or the need for a feeding tube.
9. Respiratory infections: Premature infants are at increased risk for respiratory infections, such as pneumonia and bronchiolitis.
10. Developmental delays: Premature infants may be at risk for developmental delays or learning disabilities, particularly if they experienced significant health problems or required oxygen therapy.

It is important to note that not all premature infants will develop these complications, and the severity of the conditions can vary depending on the individual baby's health and the level of care they receive. However, it is essential for parents and caregivers to be aware of the potential risks and seek prompt medical attention if they notice any signs of distress or illness in their premature infant.

Necrosis is a type of cell death that occurs when cells are exposed to excessive stress, injury, or inflammation, leading to damage to the cell membrane and the release of cellular contents into the surrounding tissue. This can lead to the formation of gangrene, which is the death of body tissue due to lack of blood supply.

There are several types of necrosis, including:

1. Coagulative necrosis: This type of necrosis occurs when there is a lack of blood supply to the tissues, leading to the formation of a firm, white plaque on the surface of the affected area.
2. Liquefactive necrosis: This type of necrosis occurs when there is an infection or inflammation that causes the death of cells and the formation of pus.
3. Caseous necrosis: This type of necrosis occurs when there is a chronic infection, such as tuberculosis, and the affected tissue becomes soft and cheese-like.
4. Fat necrosis: This type of necrosis occurs when there is trauma to fatty tissue, leading to the formation of firm, yellowish nodules.
5. Necrotizing fasciitis: This is a severe and life-threatening form of necrosis that affects the skin and underlying tissues, often as a result of bacterial infection.

The diagnosis of necrosis is typically made through a combination of physical examination, imaging studies such as X-rays or CT scans, and laboratory tests such as biopsy. Treatment depends on the underlying cause of the necrosis and may include antibiotics, surgical debridement, or amputation in severe cases.

People with Antisocial Personality Disorder may exhibit a range of symptoms, including:

* A lack of empathy or remorse for harming others
* Impulsivity and a tendency to act on whim without considering the consequences
* Aggressive or violent behavior
* A disregard for the law and a willingness to engage in criminal activity
* Difficulty forming and maintaining relationships
* Inability to feel guilt or remorse
* Inability to take responsibility for one's actions
* A tendency to manipulate others for personal gain

It is important to note that Antisocial Personality Disorder is not the same as Asperger's Syndrome or Autism Spectrum Disorder, which are separate neurodevelopmental disorders. However, people with Antisocial Personality Disorder may also have co-occurring conditions such as substance use disorders or other mental health conditions.

Treatment for Antisocial Personality Disorder typically involves a combination of psychotherapy and medication. Cognitive-behavioral therapy (CBT) and psychodynamic therapy may be effective in helping individuals with this condition to understand and change their behavior. Medications such as antidepressants and antipsychotics may also be used to help manage symptoms.

It is important to note that Antisocial Personality Disorder is a complex and challenging condition to treat, and it is not uncommon for individuals with this disorder to have difficulty adhering to treatment plans or engaging in therapy. However, with the right treatment and support, it is possible for individuals with Antisocial Personality Disorder to learn new coping skills and make positive changes in their lives.

Meningioma can occur in various locations within the brain, including the cerebrum, cerebellum, brainstem, and spinal cord. The most common type of meningioma is the meningothelial meningioma, which arises from the arachnoid membrane, one of the three layers of the meninges. Other types of meningioma include the dural-based meningioma, which originates from the dura mater, and the fibrous-cap meningioma, which is characterized by a fibrous cap covering the tumor.

The symptoms of meningioma can vary depending on the location and size of the tumor, but they often include headaches, seizures, weakness or numbness in the arms or legs, and changes in vision, memory, or cognitive function. As the tumor grows, it can compress the brain tissue and cause damage to the surrounding structures, leading to more severe symptoms such as difficulty speaking, walking, or controlling movement.

The diagnosis of meningioma typically involves a combination of imaging studies such as MRI or CT scans, and tissue sampling through biopsy or surgery. Treatment options for meningioma depend on the size, location, and aggressiveness of the tumor, but may include surgery, radiation therapy, and chemotherapy. Overall, the prognosis for meningioma is generally good, with many patients experiencing a good outcome after treatment. However, some types of meningioma can be more aggressive and difficult to treat, and the tumor may recur in some cases.

There are several types of learning disorders, including:

1. Dyslexia: A learning disorder that affects an individual's ability to read and spell words. Individuals with dyslexia may have difficulty recognizing letters, sounds, or word patterns.
2. Dyscalculia: A learning disorder that affects an individual's ability to understand and perform mathematical calculations. Individuals with dyscalculia may have difficulty with numbers, quantities, or mathematical concepts.
3. Dysgraphia: A learning disorder that affects an individual's ability to write and spell words. Individuals with dysgraphia may have difficulty with hand-eye coordination, fine motor skills, or language processing.
4. Attention Deficit Hyperactivity Disorder (ADHD): A neurodevelopmental disorder that affects an individual's ability to focus, pay attention, and regulate their behavior. Individuals with ADHD may have difficulty with organization, time management, or following instructions.
5. Auditory Processing Disorder: A learning disorder that affects an individual's ability to process and understand auditory information. Individuals with auditory processing disorder may have difficulty with listening, comprehension, or speech skills.
6. Visual Processing Disorder: A learning disorder that affects an individual's ability to process and understand visual information. Individuals with visual processing disorder may have difficulty with reading, writing, or other tasks that require visual processing.
7. Executive Function Deficits: A learning disorder that affects an individual's ability to plan, organize, and execute tasks. Individuals with executive function deficits may have difficulty with time management, organization, or self-regulation.

Learning disorders can be diagnosed by a trained professional, such as a psychologist, neuropsychologist, or learning specialist, through a comprehensive assessment that includes cognitive and academic testing, as well as a review of the individual's medical and educational history. The specific tests and assessments used will depend on the suspected type of learning disorder and the individual's age and background.

There are several approaches to treating learning disorders, including:

1. Accommodations: Providing individuals with accommodations, such as extra time to complete assignments or the option to take a test orally, can help level the playing field and enable them to succeed academically.
2. Modifications: Making modifications to the curriculum or instructional methods can help individuals with learning disorders access the material and learn in a way that is tailored to their needs.
3. Therapy: Cognitive-behavioral therapy (CBT) and other forms of therapy can help individuals with learning disorders develop strategies for managing their challenges and improving their academic performance.
4. Assistive technology: Assistive technology, such as text-to-speech software or speech-to-text software, can help individuals with learning disorders access information and communicate more effectively.
5. Medication: In some cases, medication may be prescribed to help manage symptoms associated with learning disorders, such as attention deficit hyperactivity disorder (ADHD).
6. Multi-sensory instruction: Using multiple senses (such as sight, sound, and touch) to learn new information can be helpful for individuals with learning disorders.
7. Self-accommodations: Teaching individuals with learning disorders how to identify and use their own strengths and preferences to accommodate their challenges can be effective in helping them succeed academically.
8. Parental involvement: Encouraging parents to be involved in their child's education and providing them with information and resources can help them support their child's learning and development.
9. Collaboration: Collaborating with other educators, professionals, and family members to develop a comprehensive treatment plan can help ensure that the individual receives the support they need to succeed academically.

It is important to note that each individual with a learning disorder is unique and may respond differently to different treatments. A comprehensive assessment and ongoing monitoring by a qualified professional is necessary to determine the most effective treatment plan for each individual.

Some examples of nervous system malformations include:

1. Neural tube defects: These are among the most common types of nervous system malformations and occur when the neural tube, which forms the brain and spinal cord, fails to close properly during fetal development. Examples include anencephaly (absence of a major portion of the brain), spina bifida (incomplete closure of the spine), and encephalocele (protrusion of the brain or meninges through a skull defect).
2. Cerebral palsy: This is a group of disorders that affect movement, balance, and posture, often resulting from brain damage during fetal development or early childhood. The exact cause may not be known, but it can be related to genetic mutations, infections, or other factors.
3. Hydrocephalus: This is a condition in which there is an abnormal accumulation of cerebrospinal fluid (CSF) in the brain, leading to increased pressure and enlargement of the head. It can be caused by a variety of factors, including genetic mutations, infections, or blockages in the CSF circulatory system.
4. Moyamoya disease: This is a rare condition caused by narrowing or blockage of the internal carotid artery and its branches, leading to reduced blood flow to the brain. It can result in stroke-like episodes, seizures, and cognitive impairment.
5. Spinal muscular atrophy: This is a genetic disorder that affects the nerve cells responsible for controlling voluntary muscle movement, leading to progressive muscle weakness and wasting. It can be diagnosed through blood tests or genetic analysis.
6. Neurofibromatosis: This is a genetic disorder that causes non-cancerous tumors to grow on nerve tissue, leading to symptoms such as skin changes, learning disabilities, and eye problems. It can be diagnosed through clinical evaluation and genetic testing.
7. Tuberous sclerosis: This is a rare genetic disorder that causes non-cancerous tumors to grow in the brain and other organs, leading to symptoms such as seizures, developmental delays, and skin changes. It can be diagnosed through clinical evaluation, imaging studies, and genetic testing.
8. Cerebral palsy: This is a group of disorders that affect movement, posture, and muscle tone, often resulting from brain damage sustained during fetal development or early childhood. It can be caused by a variety of factors, including premature birth, infections, and genetic mutations.
9. Down syndrome: This is a genetic disorder caused by an extra copy of chromosome 21, leading to intellectual disability, developmental delays, and physical characteristics such as a flat face and short stature. It can be diagnosed through blood tests or genetic analysis.
10. William syndrome: This is a rare genetic disorder caused by a deletion of genetic material on chromosome 7, leading to symptoms such as cardiovascular problems, growth delays, and learning disabilities. It can be diagnosed through clinical evaluation and genetic testing.

It's important to note that these are just a few examples of developmental disorders, and there are many other conditions that can affect cognitive and physical development in children. If you suspect your child may have a developmental disorder, it's important to speak with a qualified healthcare professional for an accurate diagnosis and appropriate treatment.

The different types of CNSVMs include:

1. Arteriovenous malformations (AVMs): These are abnormal connections between arteries and veins that can cause bleeding, seizures, and neurological deficits.
2. Cavernous malformations: These are abnormal collections of blood vessels that can cause seizures, headaches, and neurological deficits.
3. Capillary telangiectasia: These are small, fragile blood vessels that can cause seizures, headaches, and neurological deficits.
4. Venous malformations: These are abnormalities of the veins that can cause neurological symptoms and cosmetic deformities.

The diagnosis of CNSVMs is based on a combination of clinical presentation, imaging studies (such as MRI or CT scans), and angiography. Treatment options vary depending on the type and location of the malformation and may include observation, surgery, embolization, or radiosurgery. The prognosis for CNSVMs varies depending on the specific type and location of the malformation, as well as the severity of the symptoms. In general, early diagnosis and treatment can improve outcomes and reduce the risk of complications.

The symptoms of gait disorders, neurologic can vary depending on the underlying cause, but may include:

* Difficulty walking or standing
* Ataxia (loss of coordination)
* Spasticity (stiffness) or rigidity (inflexibility)
* Bradykinesia (slowness of movement)
* Scanning (looking for support while walking)
* Pauses or freezing during gait
* Loss of balance or poor equilibrium
* Increased risk of falling

Gait disorders, neurologic can have a significant impact on an individual's quality of life, as they may limit their ability to perform daily activities and increase their risk of falling. Treatment for these disorders typically involves a combination of physical therapy, occupational therapy, and medications to manage symptoms such as spasticity and bradykinesia. In some cases, surgery or other interventions may be necessary to address underlying causes of the gait disorder.

The condition is often caused by atherosclerosis, a buildup of plaque in the arteries, which can reduce blood flow to the brain and cause damage to the blood vessels. Other factors that can contribute to vertebrobasilar insufficiency include blood clots, high blood pressure, and certain medical conditions such as diabetes and high cholesterol.

Vertebrobasilar insufficiency is typically diagnosed through a physical examination, imaging tests such as CT or MRI scans, andDoppler ultrasound. Treatment options for the condition may include lifestyle changes such as regular exercise, a healthy diet, and stress management, as well as medications to lower blood pressure and cholesterol levels. In some cases, surgery may be necessary to improve blood flow to the brain.

It is important to note that vertebrobasilar insufficiency can be a serious condition and can lead to more severe complications such as stroke if left untreated. If you are experiencing symptoms of the condition, it is important to seek medical attention as soon as possible.

Lacunar strokes typically affect only a small area of the brain, resulting in a relatively mild stroke with few symptoms. However, they can sometimes occur in clusters, leading to more severe and widespread damage.

The most common cause of lacunar stroke is the buildup of plaque in the small blood vessels of the brain, which can narrow or block the flow of blood. Other risk factors for lacunar stroke include high blood pressure, diabetes, high cholesterol, smoking, and obesity.

Symptoms of lacunar stroke may include weakness or numbness in the face, arm, or leg on one side of the body, difficulty speaking or understanding speech, sudden vision loss, dizziness, and loss of balance or coordination.

Diagnosis of lacunar stroke is typically made through a combination of physical examination, medical history, and imaging tests such as CT or MRI scans. Treatment for lacunar stroke may include medications to dissolve blood clots, manage blood pressure, and control symptoms such as seizures or swelling. In some cases, surgery may be necessary to remove the blockage or repair the damaged blood vessel.

Overall, while lacunar stroke is a relatively rare type of stroke, it can have a significant impact on cognitive and physical functioning, and prompt medical attention is essential for effective treatment and recovery.

Delirium is a sudden change in mental status that can be caused by a variety of factors such as infection, medication, or surgery. It is characterized by confusion, disorientation, and a decreased level of consciousness. Dementia, on the other hand, is a gradual decline in mental abilities that can be caused by various underlying diseases such as Alzheimer's disease, vascular dementia, or frontotemporal dementia.

Amnestic disorders refer specifically to conditions that affect memory, such as amnesia, which is the inability to form new memories. Cognitive disorders are more general term that includes a wide range of conditions that affect cognitive functions such as attention, perception, language, and problem-solving abilities.

These conditions can be caused by various factors such as genetics, head trauma, infections, or diseases such as stroke or brain tumors. Diagnosis is typically made through a combination of medical history, physical examination, laboratory tests, and neuropsychological evaluations.

Treatment for delirium, dementia, amnestic, and cognitive disorders can vary depending on the underlying cause but may include medication, therapy, and lifestyle changes. In some cases, these conditions may be reversible with appropriate treatment, while in others, they may be irreversible.

It is important to seek medical attention if symptoms persist or worsen over time as early diagnosis and intervention can improve outcomes for individuals with these conditions.

AVMs are characterized by a tangle of abnormal blood vessels that can cause a variety of symptoms, including:

* Headaches
* Seizures
* Stroke-like episodes
* Neurological deficits such as weakness or numbness
* Vision problems
* Pain

AVMs can be diagnosed through a combination of imaging studies such as CT or MRI scans, and catheter angiography. Treatment options for AVMs include:

* Endovascular embolization, which involves using a catheter to inject materials into the abnormal blood vessels to block them off
* Surgery to remove the AVM
* Radiation therapy to shrink the AVM

The goal of treatment is to prevent bleeding, seizures, and other complications associated with AVMs. In some cases, treatment may not be necessary if the AVM is small and not causing any symptoms. However, in more severe cases, prompt treatment can significantly improve outcomes.

The most common types of CNS fungal infections include:

1. Meningitis: An inflammation of the membranes that cover the brain and spinal cord, caused by fungi such as Candida, Aspergillus, or Cryptococcus.
2. Encephalitis: An inflammation of the brain tissue itself, caused by fungi such as Histoplasma or Coccidioides.
3. Abscesses: Pocket of pus that form in the brain or spinal cord, caused by bacteria or fungi.
4. Opportunistic infections: Infections that occur in people with compromised immune systems, such as HIV/AIDS patients or those taking immunosuppressive drugs after an organ transplant.

CNS fungal infections can cause a wide range of symptoms, including headache, fever, confusion, seizures, and loss of coordination. They are typically diagnosed through a combination of physical examination, laboratory tests, and imaging studies such as CT or MRI scans.

Treatment of CNS fungal infections usually involves the use of antifungal medications, which can be administered intravenously or orally. The choice of treatment depends on the severity and location of the infection, as well as the patient's overall health status. In some cases, surgery may be necessary to drain abscesses or relieve pressure on the brain.

Prevention of CNS fungal infections is important for individuals at risk, such as those with compromised immune systems or underlying medical conditions. This includes taking antifungal medications prophylactically, avoiding exposure to fungal spores, and practicing good hygiene.

Overall, CNS fungal infections are serious and potentially life-threatening conditions that require prompt diagnosis and treatment. With appropriate management, many patients can recover fully, but delays in diagnosis and treatment can lead to poor outcomes.

Quadriplegia can be classified into two types:

1. Complete quadriplegia: This is when all four limbs are paralyzed and there is no movement or sensation below the level of the injury.
2. Incomplete quadriplegia: This is when some movement or sensation remains below the level of the injury, but not in all four limbs.

The symptoms of quadriplegia can vary depending on the underlying cause and severity of the condition. They may include:

* Loss of movement in the arms and legs
* Weakness or paralysis of the muscles in the arms and legs
* Decreased or absent sensation in the arms and legs
* Difficulty with balance and coordination
* Difficulty with walking, standing, or sitting
* Difficulty with performing daily activities such as dressing, grooming, and feeding oneself

The diagnosis of quadriplegia is typically made through a combination of physical examination, medical history, and imaging studies such as X-rays or MRIs. Treatment for quadriplegia depends on the underlying cause and may include:

* Physical therapy to improve strength and mobility
* Occupational therapy to learn new ways of performing daily activities
* Assistive devices such as braces, walkers, or wheelchairs
* Medications to manage pain, spasticity, or other symptoms
* Surgery to repair or stabilize the spinal cord or other affected areas.

Overall, quadriplegia is a severe condition that can significantly impact a person's quality of life. However, with appropriate treatment and support, many people with quadriplegia are able to lead active and fulfilling lives.

Causes:

The exact cause of SST is not well understood, but it is believed to be related to abnormal blood flow or coagulation disorders. Some possible causes include:

* Infection: Bacterial, viral, or fungal infections can cause inflammation and damage to the blood vessels in the brain, leading to the formation of a clot in the sagittal sinus.
* Trauma: Head injuries or other types of trauma can cause damage to the blood vessels in the brain, leading to the formation of a clot in the sagittal sinus.
* Genetic predisposition: Some people may be born with a genetic predisposition to develop SST.
* Cancer: Certain types of cancer, such as lymphoma or leukemia, can cause abnormal blood flow and increase the risk of SST.

Symptoms:

The symptoms of SST can vary depending on the location and size of the clot. Some common symptoms include:

* Headache: A severe headache is often the first symptom of SST, which may be accompanied by nausea and vomiting.
* Seizures: SST can cause seizures, especially in children.
* Confusion: Patients with SST may experience confusion, disorientation, and difficulty concentrating.
* Weakness or paralysis: Depending on the location of the clot, patients may experience weakness or paralysis in the arms, legs, or face.
* Vision problems: SST can cause vision problems, including blurred vision, double vision, or loss of peripheral vision.

Diagnosis:

SST is difficult to diagnose, as the symptoms can be similar to other conditions such as meningitis or stroke. A thorough physical examination and imaging studies are necessary to confirm the diagnosis. Some common diagnostic tests include:

* Computed tomography (CT) scan: A CT scan can help identify the location and size of the clot.
* Magnetic resonance imaging (MRI): An MRI can provide more detailed information about the clot and its effects on surrounding tissue.
* Doppler ultrasound: A Doppler ultrasound can help identify blood flow in the affected area.

Treatment:

The treatment of SST depends on the location and size of the clot, as well as the underlying cause. Some common treatment options include:

* Anticoagulant medications: These medications, such as heparin or warfarin, can help prevent further clotting.
* Thrombolytic medications: These medications, such as tissue plasminogen activator (tPA), can dissolve the clot.
* Surgery: In some cases, surgery may be necessary to remove the clot or repair damaged blood vessels.

Complications:

SST can have serious complications, including:

* Stroke: If the clot blocks the flow of blood to the brain, it can cause a stroke.
* Heart attack: If the clot blocks the flow of blood to the heart, it can cause a heart attack.
* Pulmonary embolism: If the clot breaks loose and travels to the lungs, it can cause a pulmonary embolism.

Prevention:

To prevent SST, it is important to take steps to reduce your risk factors. This can include:

* Controlling high blood pressure
* Controlling high cholesterol levels
* Quitting smoking
* Maintaining a healthy weight
* Exercising regularly
* Avoiding long periods of immobility, such as during long-distance travel.

Prognosis:

The prognosis for SST is generally good if the clot is detected and treated early. However, if the clot is left untreated, it can lead to serious complications and can be fatal.

Lifestyle changes:

To reduce the risk of developing SST, you can make lifestyle changes such as:

* Quitting smoking
* Maintaining a healthy weight
* Exercising regularly
* Avoiding long periods of immobility, such as during long-distance travel.

Medications:

There are several medications that can be used to treat SST, including:

* Anticoagulants, such as warfarin or heparin, which prevent the clot from growing larger and prevent new clots from forming.
* Thrombolytics, such as tissue plasminogen activator (tPA), which dissolve the clot.
* Antiplatelet agents, such as aspirin, which prevent platelets from sticking together and forming a clot.

Surgery:

In some cases, surgery may be necessary to treat SST. This can include:

* Endovascular therapy, such as angioplasty or stenting, which can be used to open up the blocked blood vessel.
* Bypass surgery, which can be used to reroute blood flow around the blocked blood vessel.

Complications:

SST can cause a number of complications, including:

* Pulmonary embolism: If a piece of the clot breaks loose and travels to the lungs, it can cause a pulmonary embolism, which can be life-threatening.
* Stroke or brain damage: If the clot blocks blood flow to the brain, it can cause a stroke or brain damage.
* Infection: The clot can become infected, which can lead to sepsis and other complications.
* Amputation: In severe cases, SST can lead to tissue death and amputation of the affected limb.

Prognosis:

The prognosis for SST is generally good if the condition is diagnosed and treated promptly. However, if left untreated, it can lead to serious complications and even death. The overall survival rate for SST is about 80%.

Lifestyle Changes:

There are several lifestyle changes that can help reduce the risk of developing SST, including:

* Maintaining a healthy weight
* Exercising regularly
* Avoiding long periods of immobility, such as during long-distance travel.

Medications:

There are several medications that can be used to treat SST, including:

* Anticoagulants, which prevent the clot from growing and prevent new clots from forming.
* Thrombolytics, which dissolve the clot.
* Antiplatelet agents, which prevent platelets from sticking together and forming a clot.

Surgery:

In some cases, surgery may be necessary to treat SST. This can include:

* Thrombectomy, which involves removing the clot.
* Embolectomy, which involves removing the clot from the blood vessel.

Conclusion:

SST is a serious condition that can lead to severe complications if left untreated. However, with prompt diagnosis and treatment, the prognosis is generally good. Lifestyle changes and medications can help reduce the risk of developing SST, and surgery may be necessary in some cases. It is important to seek medical attention immediately if you experience any symptoms of SST.

There are several causes of hemianopsia, including:

1. Stroke or cerebral vasculitis: These conditions can damage the occipital lobe and result in hemianopsia.
2. Brain tumors: Tumors in the occipital lobe can cause hemianopsia by compressing or damaging the visual pathways.
3. Traumatic brain injury: A head injury can cause damage to the occipital lobe and result in hemianopsia.
4. Cerebral palsy: This condition can cause brain damage that leads to hemianopsia.
5. Multiple sclerosis: This autoimmune disease can cause damage to the visual pathways and result in hemianopsia.

Symptoms of hemianopsia may include:

1. Blindness or impaired vision in one side of both eyes.
2. Difficulty recognizing objects or people on one side of the visual field.
3. Inability to see objects that are peripheral to the affected side.
4. Difficulty with depth perception and spatial awareness.
5. Eye movements that are abnormal or restricted.

Diagnosis of hemianopsia typically involves a comprehensive eye exam, including visual acuity testing, visual field testing, and imaging studies such as MRI or CT scans to evaluate the brain. Treatment options for hemianopsia depend on the underlying cause and may include:

1. Glasses or contact lenses to correct refractive errors.
2. Prism lenses to realign the visual image.
3. Visual therapy to improve remaining vision.
4. Medications to treat underlying conditions such as multiple sclerosis or brain tumors.
5. Surgery to repair damaged blood vessels or relieve pressure on the brain.

It is important to note that hemianopsia can significantly impact daily life and may affect an individual's ability to perform certain tasks, such as driving or reading. However, with proper diagnosis and treatment, many people with hemianopsia are able to adapt and lead fulfilling lives.

There are several types of gliomas, including:

1. Astrocytoma: This is the most common type of glioma, accounting for about 50% of all cases. It arises from the star-shaped cells called astrocytes that provide support and nutrients to the brain's nerve cells.
2. Oligodendroglioma: This type of glioma originates from the oligodendrocytes, which are responsible for producing the fatty substance called myelin that insulates the nerve fibers.
3. Glioblastoma (GBM): This is the most aggressive and malignant type of glioma, accounting for about 70% of all cases. It is fast-growing and often spreads to other parts of the brain.
4. Brain stem glioma: This type of glioma arises in the brain stem, which is responsible for controlling many of the body's vital functions such as breathing, heart rate, and blood pressure.

The symptoms of glioma depend on the location and size of the tumor. Common symptoms include headaches, seizures, weakness or numbness in the arms or legs, and changes in personality, memory, or speech.

Gliomas are diagnosed through a combination of imaging tests such as CT or MRI scans, and tissue biopsy to confirm the presence of cancer cells. Treatment options for glioma depend on the type and location of the tumor, as well as the patient's overall health. Surgery is often the first line of treatment to remove as much of the tumor as possible, followed by radiation therapy and/or chemotherapy to kill any remaining cancer cells.

The prognosis for glioma patients varies depending on the type and location of the tumor, as well as the patient's overall health. In general, the prognosis is better for patients with slow-growing, low-grade tumors, while those with fast-growing, high-grade tumors have a poorer prognosis. Overall, the 5-year survival rate for glioma patients is around 30-40%.

The term extravasation is commonly used in medical contexts to describe the leakage of fluids or medications from a blood vessel or other body structure. In the context of diagnostic and therapeutic materials, extravasation can refer to the leakage of materials such as contrast agents, medications, or other substances used for diagnostic or therapeutic purposes.

Extravagation of diagnostic and therapeutic materials can have significant consequences, including tissue damage, infection, and systemic toxicity. For example, if a contrast agent used for imaging purposes leaks into the surrounding tissues, it can cause inflammation or other adverse reactions. Similarly, if a medication intended for injection into a specific location leaks into the surrounding tissues or organs, it can cause unintended side effects or toxicity.

To prevent extravasation of diagnostic and therapeutic materials, healthcare providers must follow proper techniques and protocols for administration and use of these materials. This may include using sterile equipment, following proper injection techniques, and monitoring the patient closely for any signs of complications. In cases where extravasation does occur, prompt treatment and management are essential to minimize potential harm and prevent long-term consequences.

Dissecting aneurysms are often caused by trauma, such as a car accident or fall, but they can also be caused by other factors such as atherosclerosis (hardening of the arteries) or inherited conditions. They can occur in any blood vessel, but are most common in the aorta, which is the main artery that carries oxygenated blood from the heart to the rest of the body.

Symptoms of dissecting aneurysms can include sudden and severe pain, numbness or weakness, and difficulty speaking or understanding speech. If left untreated, a dissecting aneurysm can lead to serious complications such as stroke, heart attack, or death.

Treatment for dissecting aneurysms typically involves surgery to repair the damaged blood vessel. In some cases, endovascular procedures such as stenting or coiling may be used to treat the aneurysm. The goal of treatment is to prevent further bleeding and damage to the blood vessel, and to restore normal blood flow to the affected area.

Preventive measures for dissecting aneurysms are not always possible, but maintaining a healthy lifestyle, avoiding trauma, and managing underlying conditions such as hypertension or atherosclerosis can help reduce the risk of developing an aneurysm. Early detection and treatment are key to preventing serious complications and improving outcomes for patients with dissecting aneurysms.

There are several types of migraine disorders, including:

1. Migraine without aura: This is the most common type of migraine, characterized by a throbbing headache on one side of the head, often accompanied by sensitivity to light and sound, nausea, and vomiting.
2. Migraine with aura: This type of migraine is characterized by aura symptoms, such as visual disturbances, speech difficulties, and other neurological symptoms, which occur before the headache.
3. Chronic migraine: This type of migraine is characterized by headaches that occur 15 days or more per month, and can be accompanied by other symptoms such as fatigue, depression, and anxiety.
4. Hemiplegic migraine: This is a rare type of migraine that is characterized by a temporary weakness or paralysis on one side of the body, often accompanied by a severe headache.
5. Familial hemiplegic migraine: This is a rare inherited condition that is characterized by recurrent episodes of temporary weakness or paralysis on one side of the body, often accompanied by headaches.
6. Sporadic hemiplegic migraine: This is a rare condition that is characterized by recurrent episodes of temporary weakness or paralysis on one side of the body, often accompanied by headaches, but without a clear family history.
7. Migraine-related disorders: These are conditions that are associated with migraine, such as stroke, seizures, and autonomic dysfunction.

Migraine disorders can be difficult to diagnose, as the symptoms can vary in severity and frequency, and may overlap with other conditions. However, there are several diagnostic criteria that healthcare providers use to identify migraine disorders, including:

1. Headache frequency: Migraine headaches typically occur more frequently than headaches caused by other conditions, such as tension headaches or sinus headaches.
2. Headache severity: Migraine headaches can be severe and debilitating, often requiring bed rest or medication to relieve the pain.
3. Associated symptoms: Migraine headaches are often accompanied by other symptoms, such as sensitivity to light and sound, nausea, vomiting, and visual disturbances.
4. Family history: A family history of migraine can increase the likelihood of a diagnosis.
5. Physical examination: A healthcare provider may perform a physical examination to look for signs of migraine, such as tenderness in the head and neck muscles or changes in the sensation and strength of the limbs.
6. Imaging tests: Imaging tests, such as CT or MRI scans, may be ordered to rule out other conditions that can cause similar symptoms.
7. Medication trials: Healthcare providers may prescribe medications to treat migraine headaches and observe the patient's response to determine if the condition is migraine-related.

There are several types of headaches, including:

1. Tension headaches: These headaches are caused by muscle tension in the neck and scalp and can be treated with over-the-counter pain relievers.
2. Sinus headaches: These headaches are caused by inflammation or infection in the sinuses and can be treated with antibiotics or decongestants.
3. Cluster headaches: These headaches occur in clusters or cycles and can be very severe, often waking the patient up during the night.
4. Rebound headaches: These headaches are caused by overuse of pain medications and can be treated by stopping the medication and using alternative therapies.
5. Hormonal headaches: These headaches are related to changes in hormone levels, such as those experienced during menstruation or menopause.
6. Caffeine headaches: These headaches are caused by excessive caffeine consumption and can be treated by reducing or avoiding caffeine intake.
7. Dehydration headaches: These headaches are caused by dehydration and can be treated by drinking plenty of water.
8. Medication overuse headaches: These headaches are caused by taking too much pain medication and can be treated by stopping the medication and using alternative therapies.
9. Chronic daily headaches: These headaches are defined as headaches that occur 15 days or more per month and can be caused by a variety of factors, including muscle tension, sinus problems, and other underlying conditions.
10. Migraine headaches: These headaches are characterized by severe pain, often on one side of the head, along with other symptoms such as nausea, vomiting, and sensitivity to light and sound. They can be treated with over-the-counter or prescription medications, as well as alternative therapies such as acupuncture and relaxation techniques.

Headaches can be caused by a variety of factors, including:

1. Muscle tension: Tight muscles in the neck and scalp can lead to headaches.
2. Sinus problems: Inflammation or infection in the sinuses can cause headaches.
3. Allergies: Seasonal allergies or allergies to certain foods or substances can cause headaches.
4. Eye strain: Prolonged use of computers, smartphones, or other digital devices can cause eye strain and lead to headaches.
5. Sleep disorders: Poor sleep quality or insomnia can contribute to headaches.
6. Hormonal changes: Changes in estrogen levels, such as those experienced during menstruation or menopause, can cause headaches.
7. Dehydration: Not drinking enough water can lead to dehydration and contribute to headaches.
8. Poor posture: Slouching or hunching over can lead to muscle tension and contribute to headaches.
9. Stress: High levels of stress can cause muscle tension and contribute to headaches.
10. Diet: Certain foods, such as alcohol, caffeine, chocolate, and MSG, can trigger headaches in some people.

It is important to seek medical attention if you experience any of the following symptoms along with your headache:

1. Fever
2. Confusion or disorientation
3. Severe neck stiffness
4. Pain that worsens with movement or coughing
5. Headaches that occur more frequently or are more severe than usual
6. Headaches that interfere with daily activities or sleep
7. Sudden, severe headaches in someone who has never experienced them before
8. Headaches in someone who is taking certain medications or has a history of medical conditions such as migraines or stroke.

A healthcare professional can help determine the underlying cause of your headaches and recommend appropriate treatment options.

The symptoms of meningoencephalitis can vary depending on the cause, but common signs include fever, headache, stiff neck, confusion, seizures, and loss of consciousness. The disease can progress rapidly and can be fatal if not treated promptly.

Diagnosis is typically made through a combination of physical examination, laboratory tests (such as blood cultures and PCR), and imaging studies (such as CT or MRI scans). Treatment options depend on the underlying cause, but may include antibiotics, antiviral medications, and supportive care to manage symptoms and prevent complications.

Prognosis for meningoencephalitis depends on the severity of the disease and the promptness and effectiveness of treatment. In general, the prognosis is better for patients who receive prompt medical attention and have a mild form of the disease. However, the disease can be severe and potentially life-threatening, especially in young children, older adults, and those with weakened immune systems.

Some common types of memory disorders include:

1. Amnesia: A condition where an individual experiences memory loss, either partial or total, due to brain damage or other causes.
2. Dementia: A broad term that describes a decline in cognitive function, including memory loss, confusion, and difficulty with communication and daily activities. Alzheimer's disease is the most common cause of dementia.
3. Mild Cognitive Impairment (MCI): A condition characterized by memory loss and other cognitive symptoms that are more severe than normal age-related changes but not as severe as dementia.
4. Attention Deficit Hyperactivity Disorder (ADHD): A neurodevelopmental disorder that affects attention, impulse control, and hyperactivity. Memory problems are often a component of ADHD.
5. Traumatic Brain Injury (TBI): A condition that occurs when the brain is injured due to a blow or jolt to the head, which can result in memory loss and other cognitive problems.
6. Stroke: A condition where blood flow to the brain is interrupted, leading to brain cell death and potential memory loss.
7. Meningitis: An inflammatory condition that affects the membranes covering the brain and spinal cord, which can lead to memory loss and other cognitive problems.
8. Encephalitis: An inflammatory condition that affects the brain directly, leading to memory loss and other cognitive problems.
9. Chronic Fatigue Syndrome (CFS): A condition characterized by persistent fatigue, memory loss, and other cognitive symptoms.
10. Sleep Disorders: Sleep disturbances can affect memory and cognitive function, including conditions such as insomnia, sleep apnea, and restless leg syndrome.

The diagnosis of memory disorders typically involves a combination of medical history, physical examination, laboratory tests, and neuropsychological evaluations. The specific treatment approach will depend on the underlying cause of the memory loss, but may include medication, behavioral interventions, and lifestyle changes.

What is the meaning of intracranial hemorrhage hypertensive?

Find out what is the full meaning of INTRACRANIAL HEMORRHAGE, HYPERTENSIVE on Abbreviations.com! 'INTRACRANIAL HEMORRHAGE, HYPERTENSIVE' is an acronym for Intracranial Hemorrhage, Hypertensive.

Hypertensive intracerebral hemorrhage (HIH) is a type of bleeding in the brain caused by high blood pressure. It can lead to serious complications and even death. In this article, we will explore the causes, symptoms, diagnosis, treatment, and prevention of HIH.

Hypertensive intracerebral hemorrhage is a type of stroke that occurs when a blood vessel in the brain ruptures and bleeds into the surrounding tissue. The bleeding can cause damage to the brain tissue and lead to a variety of symptoms, including headache, confusion, weakness or numbness in the arms or legs, difficulty speaking or understanding speech, and vision problems.

Hypertensive intracerebral hemorrhage (HIH) is a type of stroke that occurs when high blood pressure causes a blood vessel in the brain to rupture and bleed into the surrounding tissue. HIH can cause serious complications and even death, so it is important to seek medical attention immediately if symptoms persist or worsen over time.

Hypertensive intracerebral hemorrhage (HIH) is a type of stroke that occurs when high blood pressure causes a blood vessel in the brain to rupture and bleed into the surrounding tissue. This can lead to serious complications, such as brain damage, seizures, and even death.

Hypertensive intracerebral hemorrhage (HIH) is a type of stroke that occurs when high blood pressure causes a blood vessel in the brain to rupture and bleed into the surrounding tissue. It can cause serious complications, such as brain damage, seizures, and even death.

Hypertensive intracerebral hemorrhage (HIH) is a type of stroke that occurs when high blood pressure causes a blood vessel in the brain to rupture and bleed into the surrounding tissue. This can lead to serious complications, such as brain damage, seizures, and even death. Treatment for HIH typically involves controlling blood pressure and managing any related symptoms.

Hypertensive intracerebral hemorrhage (HIH) is a type of stroke that occurs when high blood pressure causes a blood vessel in the brain to rupture and bleed into the surrounding tissue. Treatment for HIH typically involves controlling blood pressure and managing any related symptoms, such as seizures or brain damage.

Hypertensive intracerebral hemorrhage (HIH) is a type of stroke that occurs when high blood pressure causes a blood vessel in the brain to rupture and bleed into the surrounding tissue. Treatment for HIH typically involves controlling blood pressure and managing any related symptoms, such as seizures or brain damage. In some cases, surgery may be necessary to relieve pressure on the affected area of the brain.

Hypertensive intracerebral hemorrhage (HIH) is a type of stroke that occurs when high blood pressure causes a blood vessel in the brain to rupture and bleed into the surrounding tissue. Treatment for HIH typically involves controlling blood pressure and managing any related symptoms, such as seizures or brain damage. In some cases, surgery may be necessary to relieve pressure on the affected area of the brain. Additionally, medications such as anticonvulsants and vasospasmolytics may be used to manage seizures and reduce the risk of further complications.

The word "athetosis" comes from the Greek words "a-" meaning "without" and "thesis" meaning "position" or "movement". In the context of medicine, the term is used to describe a condition where there is an absence of normal movement patterns, resulting in slow and writhing movements.

The symptoms of athetosis can vary in severity and may include:

1. Slow, writhing movements of the hands and feet
2. Lack of coordination and balance
3. Stiffness and rigidity in the muscles
4. Difficulty with walking and standing
5. Weakness in the limbs

Athetosis can be diagnosed through a combination of physical examination, medical history, and imaging tests such as MRI or CT scans. There is no cure for athetosis, but treatment options may include physical therapy, occupational therapy, and medications to manage symptoms.

In summary, athetosis is a movement disorder characterized by slow, writhing movements of the hands and feet, often associated with other conditions such as cerebral palsy or Down syndrome. It can be diagnosed through physical examination and imaging tests, and treated with a combination of therapies and medications.

There are several types of amyloidosis, each with different causes and symptoms. The most common types include:

1. Primary amyloidosis: This type is caused by the production of abnormal proteins in the bone marrow. It mainly affects older adults and can lead to symptoms such as fatigue, weight loss, and numbness or tingling in the hands and feet.
2. Secondary amyloidosis: This type is caused by other conditions, such as rheumatoid arthritis, tuberculosis, or inflammatory bowel disease. It can also be caused by long-term use of certain medications, such as antibiotics or chemotherapy.
3. Familial amyloid polyneuropathy: This type is inherited and affects the nerves in the body, leading to symptoms such as muscle weakness, numbness, and pain.
4. Localized amyloidosis: This type affects a specific area of the body, such as the tongue or the skin.

The symptoms of amyloidosis can vary depending on the organs affected and the severity of the condition. Some common symptoms include:

1. Fatigue
2. Weakness
3. Pain
4. Numbness or tingling in the hands and feet
5. Swelling in the legs, ankles, and feet
6. Difficulty with speech or swallowing
7. Seizures
8. Heart problems
9. Kidney failure
10. Liver failure

The diagnosis of amyloidosis is based on a combination of physical examination, medical history, laboratory tests, and imaging studies. Laboratory tests may include blood tests to measure the levels of certain proteins in the body, as well as biopsies to examine tissue samples under a microscope. Imaging studies, such as X-rays, CT scans, and MRI scans, may be used to evaluate the organs affected by the condition.

There is no cure for amyloidosis, but treatment can help manage the symptoms and slow the progression of the disease. Treatment options may include:

1. Medications to control symptoms such as pain, swelling, and heart problems
2. Chemotherapy to reduce the production of abnormal proteins
3. Autologous stem cell transplantation to replace damaged cells with healthy ones
4. Dialysis to remove excess fluids and waste products from the body
5. Nutritional support to ensure adequate nutrition and hydration
6. Physical therapy to maintain muscle strength and mobility
7. Supportive care to manage pain, improve quality of life, and reduce stress on the family.

In conclusion, amyloidosis is a complex and rare group of diseases that can affect multiple organs and systems in the body. Early diagnosis and treatment are essential to managing the symptoms and slowing the progression of the disease. It is important for patients with suspected amyloidosis to seek medical attention from a specialist, such as a hematologist or nephrologist, for proper evaluation and treatment.

There are several key features of inflammation:

1. Increased blood flow: Blood vessels in the affected area dilate, allowing more blood to flow into the tissue and bringing with it immune cells, nutrients, and other signaling molecules.
2. Leukocyte migration: White blood cells, such as neutrophils and monocytes, migrate towards the site of inflammation in response to chemical signals.
3. Release of mediators: Inflammatory mediators, such as cytokines and chemokines, are released by immune cells and other cells in the affected tissue. These molecules help to coordinate the immune response and attract more immune cells to the site of inflammation.
4. Activation of immune cells: Immune cells, such as macrophages and T cells, become activated and start to phagocytose (engulf) pathogens or damaged tissue.
5. Increased heat production: Inflammation can cause an increase in metabolic activity in the affected tissue, leading to increased heat production.
6. Redness and swelling: Increased blood flow and leakiness of blood vessels can cause redness and swelling in the affected area.
7. Pain: Inflammation can cause pain through the activation of nociceptors (pain-sensing neurons) and the release of pro-inflammatory mediators.

Inflammation can be acute or chronic. Acute inflammation is a short-term response to injury or infection, which helps to resolve the issue quickly. Chronic inflammation is a long-term response that can cause ongoing damage and diseases such as arthritis, asthma, and cancer.

There are several types of inflammation, including:

1. Acute inflammation: A short-term response to injury or infection.
2. Chronic inflammation: A long-term response that can cause ongoing damage and diseases.
3. Autoimmune inflammation: An inappropriate immune response against the body's own tissues.
4. Allergic inflammation: An immune response to a harmless substance, such as pollen or dust mites.
5. Parasitic inflammation: An immune response to parasites, such as worms or fungi.
6. Bacterial inflammation: An immune response to bacteria.
7. Viral inflammation: An immune response to viruses.
8. Fungal inflammation: An immune response to fungi.

There are several ways to reduce inflammation, including:

1. Medications such as nonsteroidal anti-inflammatory drugs (NSAIDs), corticosteroids, and disease-modifying anti-rheumatic drugs (DMARDs).
2. Lifestyle changes, such as a healthy diet, regular exercise, stress management, and getting enough sleep.
3. Alternative therapies, such as acupuncture, herbal supplements, and mind-body practices.
4. Addressing underlying conditions, such as hormonal imbalances, gut health issues, and chronic infections.
5. Using anti-inflammatory compounds found in certain foods, such as omega-3 fatty acids, turmeric, and ginger.

It's important to note that chronic inflammation can lead to a range of health problems, including:

1. Arthritis
2. Diabetes
3. Heart disease
4. Cancer
5. Alzheimer's disease
6. Parkinson's disease
7. Autoimmune disorders, such as lupus and rheumatoid arthritis.

Therefore, it's important to manage inflammation effectively to prevent these complications and improve overall health and well-being.

Hyperoxia can cause damage to the body's tissues and organs, particularly the lungs and brain. In severe cases, hyperoxia can lead to respiratory failure, seizures, and even death.

There are several ways to diagnose hyperoxia, including:

1. Blood tests: These can measure the levels of oxygen in the blood.
2. Arterial blood gas (ABG) analysis: This is a test that measures the amounts of oxygen and carbon dioxide in the blood.
3. Pulse oximetry: This is a non-invasive test that measures the amount of oxygen in the blood by shining a light through the skin.

Treatment for hyperoxia depends on the underlying cause, but may include:

1. Oxygen therapy: This involves administering oxygen to the patient through a mask or nasal tubes.
2. Medications: These may be used to treat any underlying conditions that are causing hyperoxia.
3. Mechanical ventilation: In severe cases, this may be necessary to support the patient's breathing.

In summary, hyperoxia is a condition where there is too much oxygen in the body, and it can cause damage to the body's tissues and organs. Diagnosis is typically made through blood tests or other tests, and treatment may involve oxygen therapy, medications, or mechanical ventilation.

The symptoms of sparganosis can vary depending on the location of the infection, but may include:

* Abdominal pain
* Diarrhea
* Nausea and vomiting
* Skin rashes or lesions
* Eye inflammation
* Meningitis

Sparganosis is diagnosed through a combination of physical examination, medical history, and laboratory tests such as blood tests and imaging studies. Treatment typically involves surgical removal of the parasitic larvae and supportive care to manage symptoms. In severe cases, hospitalization may be necessary to monitor and treat complications.

Prevention measures for sparganosis include:

* Avoiding consumption of raw or undercooked meat of aquatic animals
* Properly cooking fish and frogs before eating them
* Avoiding contact with water that may be contaminated with parasites
* Practicing good hygiene, such as washing hands after handling raw meat.

Some common causes of confusion in the medical field include:

1. Medication side effects: Certain medications can cause confusion as a side effect, particularly those that affect the central nervous system, such as sedatives, anti-anxiety drugs, and painkillers.
2. Delirium: A sudden and severe confusional state that can be caused by various factors, such as infections, surgery, or alcohol or drug withdrawal.
3. Dementia: A progressive decline in cognitive function that can cause confusion, memory loss, and difficulty with communication and daily activities. Alzheimer's disease is the most common cause of dementia.
4. Traumatic brain injury: A head injury that can cause confusion, memory loss, and other cognitive difficulties.
5. Stroke: A loss of blood flow to the brain that can cause confusion, weakness, and difficulty with speech and mobility.
6. Infections: Certain infections, such as urinary tract infections or sepsis, can cause confusion and disorientation.
7. Sleep disorders: Sleep disturbances, such as insomnia or sleep apnea, can lead to confusion and other cognitive difficulties.
8. Chronic conditions: Certain chronic conditions, such as diabetes, hypertension, or hypothyroidism, can cause confusion if left untreated or poorly managed.
9. Nutritional deficiencies: Deficiencies in certain nutrients, such as vitamin B12 or thiamine, can cause confusion and other cognitive difficulties.
10. Substance abuse: Withdrawal from drugs or alcohol can cause confusion, agitation, and other cognitive difficulties.

In the medical field, confusion is often evaluated through a series of questions and physical examinations to determine its underlying cause. Treatment may involve addressing any underlying conditions, managing symptoms, and providing supportive care to help improve cognitive function. In some cases, medication or other interventions may be necessary to manage symptoms and improve quality of life.

Types of Malformations of Cortical Development:

There are several types of malformations of cortical development, including:

1. Cerebral palsy: a group of disorders that affect movement, balance, and posture, often resulting from brain damage during fetal development or birth.
2. Hydrocephalus: a condition in which there is an abnormal accumulation of cerebrospinal fluid (CSF) in the brain, leading to increased intracranial pressure and enlargement of the head.
3. Microcephaly: a condition in which the brain and skull are smaller than normal, often resulting in developmental delays, intellectual disability, and seizures.
4. Macrocephaly: a condition in which the brain and skull are larger than normal, often resulting from an overproduction of CSF or a brain tumor.
5. Cortical dysplasia: a condition in which there is abnormal development of the cerebral cortex, leading to problems with movement, cognition, and behavior.
6. Fetal alcohol spectrum disorders (FASD): a group of conditions that result from exposure to alcohol during fetal development, often causing malformations of the cerebral cortex and other brain structures.
7. Genetic mutations: some genetic mutations can lead to malformations of cortical development, such as those caused by maternal infection or exposure to certain medications.
8. Infections during pregnancy: certain infections, such as rubella or toxoplasmosis, can cause malformations of cortical development if contracted during pregnancy.
9. Traumatic brain injury: a head injury during fetal development or early childhood can disrupt normal cortical development and lead to developmental delays and cognitive impairments.
10. Exposure to toxins: exposure to certain toxins, such as lead or pesticides, during fetal development can damage the developing brain and result in malformations of cortical development.

These are just a few examples of conditions that can cause malformations of cortical development. It's important to note that many of these conditions can be diagnosed through imaging studies such as MRI or CT scans, and some may require specialized testing or monitoring throughout childhood. Early detection and intervention can help improve outcomes for children with these conditions.

The parasite forms cysts in various organs of the body, including the brain, liver, lungs, and muscles. Symptoms of cysticercosis can vary depending on the location and size of the cysts, and may include seizures, headaches, vision problems, and movement disorders.

Diagnosis of cysticercosis is typically made through a combination of physical examination, imaging studies such as CT or MRI scans, and laboratory tests to detect the presence of antibodies or parasitic elements in the body. Treatment generally involves surgical removal of the cysts, and may also involve antiparasitic drugs to kill any remaining parasites.

In some cases, cysticercosis can lead to serious complications such as inflammation of the brain (meningitis) or blockage of blood vessels, which can be life-threatening. Therefore, early diagnosis and treatment are essential to prevent these complications and improve outcomes for patients with this condition.

Overall, cysticercosis is a significant health problem in many parts of the world, particularly in areas where sanitation and hygiene are poor, and can have serious consequences if left untreated.

1. Neurodegenerative diseases: These are diseases that cause progressive loss of brain cells, leading to cognitive decline and motor dysfunction. Examples include Alzheimer's disease, Parkinson's disease, and Huntington's disease.
2. Stroke: A stroke occurs when blood flow to the brain is interrupted, leading to cell death and potential long-term disability.
3. Traumatic brain injury: This type of injury occurs when the brain is subjected to a sudden and forceful impact, such as in a car accident or fall.
4. Infections: Bacterial, viral, and fungal infections can all cause CNS diseases, such as meningitis and encephalitis.
5. Autoimmune disorders: These are conditions in which the immune system mistakenly attacks healthy cells in the brain, leading to inflammation and damage. Examples include multiple sclerosis and lupus.
6. Brain tumors: Tumors can occur in any part of the brain and can be benign or malignant.
7. Cerebrovascular diseases: These are conditions that affect the blood vessels in the brain, such as aneurysms and arteriovenous malformations (AVMs).
8. Neurodevelopmental disorders: These are conditions that affect the development of the brain and nervous system, such as autism spectrum disorder and attention deficit hyperactivity disorder (ADHD).

CNS diseases can have a significant impact on quality of life, and some can be fatal. Treatment options vary depending on the specific diagnosis and severity of the disease. Some CNS diseases can be managed with medication, while others may require surgery or other interventions.

There are several different types of unconsciousness, including:

1. Concussion: A mild form of traumatic brain injury that can cause temporary unconsciousness, confusion, and amnesia.
2. Coma: A more severe form of unconsciousness that can be caused by a head injury, stroke, or other medical condition. Comas can last for days, weeks, or even months.
3. Vegetative state: A condition in which a person is unaware and unresponsive, but still has some reflexes. This can be caused by a traumatic brain injury, stroke, or other medical condition.
4. Persistent vegetative state (PVS): A long-term version of the vegetative state that can last for months or years.
5. Brain death: A permanent form of unconsciousness that is caused by severe damage to the brain.

Unconsciousness can be diagnosed through a variety of medical tests, including:

1. Neurological exam: A doctor will check the patient's reflexes, muscle strength, and sensation to determine the extent of any brain damage.
2. Imaging tests: CT or MRI scans can help doctors identify any structural abnormalities in the brain that may be causing unconsciousness.
3. Electroencephalogram (EEG): A test that measures electrical activity in the brain to determine if there is any abnormal brain wave activity.
4. Blood tests: To rule out other medical conditions that may be causing unconsciousness, such as infections or poisoning.

Treatment for unconsciousness depends on the underlying cause and can range from simple observation to complex surgical procedures. Some common treatments include:

1. Medications: To control seizures, reduce inflammation, or regulate brain activity.
2. Surgery: To relieve pressure on the brain, repair damaged blood vessels, or remove tumors.
3. Rehabilitation: To help the patient regain lost cognitive and motor function.
4. Supportive care: To address any other medical conditions that may be contributing to the unconsciousness, such as infections or respiratory failure.

The following are some common types of motor skill disorders:

1. Dyspraxia: This is a developmental condition that affects the ability to plan and perform movements. Individuals with dyspraxia may have difficulty with coordination, balance, and spatial awareness.
2. Apraxia: This is a neurological disorder that affects an individual's ability to perform voluntary movements despite having the physical strength and coordination to do so.
3. Ataxia: This is a condition that affects an individual's balance, coordination, and ability to perform purposeful movements. It can be caused by injury or disease to the cerebellum or other parts of the brain.
4. Parkinson's disease: This is a neurodegenerative disorder that affects movement, including fine motor skills such as writing and gross motor skills such as walking and balance.
5. Cerebral palsy: This is a developmental condition that can affect an individual's ability to move and control their body. It can impact both fine and gross motor skills.
6. Stroke: A stroke occurs when the blood supply to the brain is interrupted, leading to damage to the brain tissue. This can result in difficulty with movement, including fine and gross motor skills.
7. Traumatic brain injury: This occurs when the brain is injured as a result of a blow or jolt to the head. It can lead to difficulties with movement, memory, and other cognitive functions.
8. Spinal cord injury: This occurs when the spinal cord is damaged, either from trauma or disease. It can result in loss of movement and sensation below the level of the injury.
9. Multiple sclerosis: This is a chronic autoimmune disease that affects the central nervous system, including the brain and spinal cord. It can cause difficulties with movement, balance, and coordination.
10. Spina bifida: This is a congenital condition in which the spine does not properly close during fetal development. It can result in a range of physical and cognitive disabilities, including difficulty with movement and coordination.

It's important to note that these conditions can have varying levels of severity and impact on an individual's ability to move and control their body. Additionally, there are many other conditions and diseases that can affect the nervous system and result in difficulties with movement.

The symptoms of ALD usually become apparent in childhood or adolescence and can vary in severity. They may include:

* Adrenal insufficiency (a decrease in the production of hormones by the adrenal glands)
* Seizures
* Vision loss
* Cognitive decline
* Behavioral changes
* Muscle weakness and wasting

ALD is an X-linked disorder, meaning that it is more common in males than in females. Females can be carriers of the mutated gene, but they typically do not develop symptoms themselves.

There is no cure for ALD, but treatment options are available to manage the symptoms and slow the progression of the disease. These may include:

* Steroids to replace adrenal hormones
* Anticonvulsants to control seizures
* Physical therapy to maintain muscle strength and mobility
* Dietary changes to reduce fat intake and improve nutrition

Bone marrow transplantation has also been explored as a potential treatment for ALD, but the results are still uncertain.

The diagnosis of ALD is based on a combination of clinical findings, laboratory tests, and genetic analysis. Laboratory tests may include:

* Measurement of very long-chain fatty acids in the blood and cerebrospinal fluid
* Genetic testing to identify the mutation in the ABCD1 gene

The prognosis for ALD is generally poor, and the disease can be fatal within a few years of onset. However, with appropriate treatment and management, some individuals with ALD may experience a slowing of the disease progression and an improvement in their quality of life.

The diagnosis of PVS is made by a team of healthcare professionals, including neurosurgeons, neurologists, and rehabilitation specialists. The diagnosis is based on a combination of clinical examination and medical imaging studies, such as electroencephalograms (EEGs) and functional magnetic resonance imaging (fMRI).

There are three main criteria for diagnosing PVS:

1. Lack of awareness: The patient is unable to open their eyes or respond purposefully to stimuli.
2. Lack of purposeful movement: The patient is unable to move voluntarily, except for possibly some reflex movements.
3. Abnormal sleep-wake cycle: The patient exhibits a persistent vegetative state sleep-wake cycle, characterized by periods of sleep and wakefulness that are not consistent with normal sleep patterns.

Treatment for PVS is focused on supporting the patient's basic needs, such as breathing and nutrition, and managing any underlying medical conditions. However, there is no cure for PVS, and the condition is often permanent. Some patients may eventually recover some cognitive and behavioral functions over time, but many will remain in a state of persistent vegetative state for the rest of their lives.

In summary, the definition of persistent vegetative state (PVS) in the medical field refers to a severe loss of cognitive and behavioral function, resulting from traumatic brain injury or other causes, characterized by a lack of awareness, purposeful movement, and abnormal sleep-wake cycle. The diagnosis is made by a team of healthcare professionals, and treatment focuses on supporting the patient's basic needs and managing any underlying medical conditions.

The term "agenesis" refers to the failure of a structure to develop properly during fetal development. The corpus callosum is one of the largest white matter structures in the brain and plays a critical role in integrating sensory, motor, and cognitive information from both hemispheres.

Agenesis of Corpus Callosum can be caused by various genetic or environmental factors, such as:

1. Genetic mutations or deletions
2. Fetal exposure to certain drugs or infections during pregnancy
3. Maternal diabetes or other metabolic disorders
4. Trauma during pregnancy or childbirth
5. Brain injury or infection during early childhood.

Symptoms of Agenesis of Corpus Callosum can vary depending on the severity and location of the agenesis, but may include:

1. Delayed development of motor skills such as sitting, standing, and walking
2. Difficulty with language processing and speech articulation
3. Poor coordination and balance
4. Seizures or other neurological problems
5. Intellectual disability or developmental delays
6. Behavioral problems such as anxiety, depression, or autism spectrum disorder.

Diagnosis of Agenesis of Corpus Callosum typically involves a combination of physical examination, imaging studies such as MRI or CT scans, and genetic testing. Treatment for the condition may involve a multidisciplinary approach, including physical therapy, speech therapy, occupational therapy, and medication to control seizures or other symptoms. In some cases, surgery may be necessary to relieve pressure on the brain or to correct anatomical abnormalities.

Prognosis for individuals with Agenesis of Corpus Callosum varies depending on the severity of the condition and the presence of any additional health problems. However, early diagnosis and intervention can significantly improve outcomes and quality of life for these individuals. With appropriate treatment and support, many individuals with Agenesis of Corpus Callosum are able to lead fulfilling lives and achieve their goals.

Examples of fetal diseases include:

1. Down syndrome: A genetic disorder caused by an extra copy of chromosome 21, which can cause delays in physical and intellectual development, as well as increased risk of heart defects and other health problems.
2. Spina bifida: A birth defect that affects the development of the spine and brain, resulting in a range of symptoms from mild to severe.
3. Cystic fibrosis: A genetic disorder that affects the respiratory and digestive systems, causing thick mucus buildup and recurring lung infections.
4. Anencephaly: A condition where a portion of the brain and skull are missing, which is usually fatal within a few days or weeks of birth.
5. Clubfoot: A deformity of the foot and ankle that can be treated with casts or surgery.
6. Hirschsprung's disease: A condition where the nerve cells that control bowel movements are missing, leading to constipation and other symptoms.
7. Diaphragmatic hernia: A birth defect that occurs when there is a hole in the diaphragm, allowing organs from the abdomen to move into the chest cavity.
8. Gastroschisis: A birth defect where the intestines protrude through a opening in the abdominal wall.
9. Congenital heart disease: Heart defects that are present at birth, such as holes in the heart or narrowed blood vessels.
10. Neural tube defects: Defects that affect the brain and spine, such as spina bifida and anencephaly.

Early detection and diagnosis of fetal diseases can be crucial for ensuring proper medical care and improving outcomes for affected babies. Prenatal testing, such as ultrasound and blood tests, can help identify fetal anomalies and genetic disorders during pregnancy.

There are several types of asphyxia, including:

1. Respiratory asphyxia: This occurs when the individual's respiratory system is unable to provide enough oxygen to the body due to obstruction or paralysis of the respiratory muscles.
2. Cardiac asphyxia: This occurs when the heart is unable to pump enough blood to the body, leading to a lack of oxygen and nutrients.
3. Cerebral asphyxia: This occurs when the brain does not receive enough oxygen, leading to impaired consciousness, confusion, seizures, and even death.
4. Hypoxic-ischemic asphyxia: This occurs when there is a lack of oxygen and blood flow to the body's tissues, leading to tissue damage and cell death.

Asphyxia can cause a range of symptoms depending on its severity and duration, including:

1. Difficulty breathing or shortness of breath
2. Confusion, disorientation, or loss of consciousness
3. Slurred speech or inability to speak
4. Seizures or convulsions
5. Pale or blue-tinged skin
6. Low blood pressure
7. Slow heart rate
8. Decreased level of consciousness

Treatment for asphyxia depends on the underlying cause and the severity of the condition. In mild cases, treatment may involve providing oxygen therapy, administering medications to stimulate breathing, or performing other respiratory support measures. In severe cases, hospitalization may be necessary, and treatment may involve mechanical ventilation or other life-saving interventions.

Prevention of asphyxia is essential, and it can be achieved by avoiding situations that can lead to respiratory distress, such as smoking, alcohol consumption, and exposure to toxic substances. It is also important to ensure proper ventilation in enclosed spaces and to use appropriate safety equipment when working with hazardous materials or in confined areas.

In conclusion, asphyxia is a serious condition that can lead to tissue damage and cell death due to a lack of oxygen and blood flow. Prompt recognition and treatment are essential to prevent long-term brain damage and death. Prevention measures include avoiding situations that can lead to respiratory distress and ensuring proper ventilation in enclosed spaces.

There are several possible causes of hyperglycemia, including:

1. Diabetes: This is a chronic condition where the body either does not produce enough insulin or cannot use insulin effectively.
2. Insulin resistance: This occurs when the body's cells become less responsive to insulin, leading to high blood sugar levels.
3. Pancreatitis: This is inflammation of the pancreas, which can lead to high blood sugar levels.
4. Cushing's syndrome: This is a rare hormonal disorder that can cause high blood sugar levels.
5. Medications: Certain medications, such as steroids and some types of antidepressants, can raise blood sugar levels.
6. Stress: Stress can cause the release of hormones such as cortisol and adrenaline, which can raise blood sugar levels.
7. Infections: Certain infections, such as pneumonia or urinary tract infections, can cause high blood sugar levels.
8. Trauma: Traumatic injuries can cause high blood sugar levels due to the release of stress hormones.
9. Surgery: Some types of surgery, such as heart bypass surgery, can cause high blood sugar levels.
10. Pregnancy: High blood sugar levels can occur during pregnancy, especially in women who have a history of gestational diabetes.

Hyperglycemia can cause a range of symptoms, including:

1. Increased thirst and urination
2. Fatigue
3. Blurred vision
4. Headaches
5. Cuts or bruises that are slow to heal
6. Tingling or numbness in the hands and feet
7. Dry, itchy skin
8. Flu-like symptoms, such as weakness, dizziness, and stomach pain
9. Recurring skin, gum, or bladder infections
10. Sexual dysfunction in men and women

If left untreated, hyperglycemia can lead to serious complications, including:

1. Diabetic ketoacidosis (DKA): A life-threatening condition that occurs when the body produces high levels of ketones, which are acidic substances that can cause confusion, nausea, and vomiting.
2. Hypoglycemia: Low blood sugar levels that can cause dizziness, confusion, and even loss of consciousness.
3. Nerve damage: High blood sugar levels over an extended period can damage the nerves, leading to numbness, tingling, and pain in the hands and feet.
4. Kidney damage: The kidneys may become overworked and damaged if they are unable to filter out the excess glucose in the blood.
5. Eye damage: High blood sugar levels can cause damage to the blood vessels in the eyes, leading to vision loss and blindness.
6. Cardiovascular disease: Hyperglycemia can increase the risk of cardiovascular disease, including heart attacks, strokes, and peripheral artery disease.
7. Cognitive impairment: Hyperglycemia has been linked to cognitive impairment and an increased risk of dementia.

It is essential to manage hyperglycemia by making lifestyle changes, such as following a healthy diet, regular exercise, and taking medication if prescribed by a healthcare professional. Monitoring blood sugar levels regularly can help identify the signs of hyperglycemia and prevent long-term complications.

Some examples of basal ganglia diseases include:

1. Parkinson's disease: A neurodegenerative disorder characterized by tremors, rigidity, bradykinesia (slow movement), and postural instability.
2. Huntington's disease: An autosomal dominant disorder that causes progressive degeneration of the basal ganglia and a decline in cognitive, motor, and psychiatric functions.
3. Dystonia: A movement disorder characterized by sustained or intermittent muscle contractions that cause abnormal postures or movements.
4. Tourette's syndrome: A neurodevelopmental disorder characterized by multiple motor tics and at least one vocal tic, such as repeated sounds or words.
5. Obsessive-compulsive disorder (OCD): An anxiety disorder characterized by recurring thoughts or compulsions to perform repetitive behaviors.
6. Schizophrenia: A psychotic disorder characterized by hallucinations, delusions, and cognitive impairments.
7. Kleine-Levin syndrome: A rare sleep disorder characterized by recurring periods of excessive sleepiness and automatic behaviors.
8. Wilson's disease: A rare genetic disorder caused by copper accumulation in the basal ganglia, leading to cognitive and motor impairments.
9. Hemiballism: A rare movement disorder characterized by unilateral or bilateral involuntary movements of the upper limbs.
10. Chorea-acanthocytosis: A rare genetic disorder characterized by chorea (involuntary movements), acanthocytosis (abnormal red blood cell shape), and cognitive decline.

These conditions are often challenging to diagnose and manage, and may require a comprehensive evaluation by a multidisciplinary team of healthcare professionals, including neurologists, psychiatrists, geneticists, and other specialists. Early diagnosis and appropriate treatment can help improve outcomes for individuals with these conditions.

Hypothermia can be mild, moderate, or severe. Mild hypothermia is characterized by shivering and a body temperature of 95 to 97 degrees Fahrenheit (32 to 36.1 degrees Celsius). Moderate hypothermia has a body temperature of 82 to 94 degrees Fahrenheit (28 to 34 degrees Celsius), and the person may appear lethargic, drowsy, or confused. Severe hypothermia is characterized by a body temperature below 82 degrees Fahrenheit (28 degrees Celsius) and can lead to coma and even death if not treated promptly.

Treatment for hypothermia typically involves warming the person up slowly, using blankets or heating pads, and providing warm fluids to drink. In severe cases, medical professionals may use a specialized warm water bath or apply warm packs to specific areas of the body.

Preventing hypothermia is important, especially in cold weather conditions. This can be done by dressing appropriately for the weather, staying dry and avoiding wet clothing, eating regularly to maintain energy levels, and seeking shelter if you become stranded or lost. It's also essential to recognize the signs of hypothermia early on so that treatment can begin promptly.

Symptoms of meningitis may include fever, headache, stiff neck, confusion, nausea and vomiting, and sensitivity to light. In severe cases, it can lead to seizures, brain damage, and even death.

There are several types of meningitis, including:

1. Viral meningitis: This is the most common form of the infection and is usually caused by enteroviruses or herpesviruses. It is typically less severe than bacterial meningitis and resolves on its own with supportive care.
2. Bacterial meningitis: This is a more serious form of the infection and can be caused by a variety of bacteria, such as Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae. It requires prompt antibiotic treatment to prevent long-term complications and death.
3. Fungal meningitis: This type of meningitis is more common in people with weakened immune systems and is caused by fungi that are commonly found in the environment. It can be treated with antifungal medications.
4. Parasitic meningitis: This type of meningitis is rare and is caused by parasites that are typically found in tropical regions. It can be treated with antiparasitic medications.

Diagnosis of meningitis is based on a combination of clinical findings, laboratory tests, and imaging studies. Laboratory tests may include blood cultures, polymerase chain reaction (PCR) testing, and cerebrospinal fluid (CSF) analysis. Imaging studies, such as CT or MRI scans, may be used to rule out other conditions and to evaluate the extent of brain damage.

Treatment of meningitis depends on the cause of the infection and may include antibiotics, antiviral medications, antifungal medications, or supportive care to manage symptoms and prevent complications. Supportive care may include intravenous fluids, oxygen therapy, and pain management. In severe cases, meningitis may require hospitalization in an intensive care unit (ICU) and may result in long-term consequences such as hearing loss, learning disabilities, or cognitive impairment.

Prevention of meningitis includes vaccination against the bacteria or viruses that can cause the infection, good hygiene practices, and avoiding close contact with people who are sick. Vaccines are available for certain types of meningitis, such as the meningococcal conjugate vaccine (MenACWY) and the pneumococcal conjugate vaccine (PCV). Good hygiene practices include washing hands frequently, covering the mouth and nose when coughing or sneezing, and avoiding sharing food, drinks, or personal items.

In conclusion, meningitis is a serious and potentially life-threatening infection that can affect people of all ages. Early diagnosis and treatment are crucial to prevent long-term consequences and improve outcomes. Prevention includes vaccination, good hygiene practices, and avoiding close contact with people who are sick.



Arteritis can lead to a range of symptoms including fever, fatigue, joint pain, skin rashes, and difficulty speaking or swallowing. In severe cases, it can also cause cardiovascular complications such as heart attack, stroke, or organ failure.

There are several types of arteritis, each with different causes and symptoms. Some common forms of arteritis include:

1. Giant cell arteritis (GCA): This is the most common form of arteritis and primarily affects older adults. It is caused by inflammation of the medium-sized arteries, particularly those in the head and neck. Symptoms may include headaches, vision loss, and pain in the face and jaw.
2. Takayasu arteritis (TA): This is a rare form of arteritis that affects the aorta and its branches. It is more common in women than men and typically affects young adults. Symptoms may include high blood pressure, chest pain, and weakness or numbness in the limbs.
3. Polyarteritis nodosa (PAN): This is a rare form of arteritis that affects multiple arteries throughout the body. It can cause symptoms such as fever, fatigue, joint pain, and skin rashes.
4. Kawasaki disease: This is a rare inflammatory disease that primarily affects children under the age of 5. It causes inflammation in the blood vessels, particularly those in the heart and can lead to cardiovascular complications if left untreated.

Arteritis can be diagnosed through various tests such as blood tests, imaging studies like CT or MRI scans, and biopsies. Treatment options vary depending on the type of arteritis and its severity but may include corticosteroids, immunosuppressive medications, and antibiotics. Early diagnosis and treatment are crucial to prevent long-term damage and improve outcomes.

Some common types of intracranial arterial diseases include:

1. Atherosclerosis: The buildup of plaque in the walls of the intracranial arteries, which can narrow or block the flow of blood to the brain.
2. Moyamoya disease: A rare condition caused by narrowing or blockage of the internal carotid artery and its branches, leading to decreased blood flow to the brain.
3. Intracranial aneurysms: Weaknesses in the walls of the intracranial arteries that can lead to ballooning and potentially rupture, causing bleeding in the brain.
4. Arteriovenous malformations (AVMs): Abnormal connections between arteries and veins in the brain that can cause bleeding, seizures, and other neurological symptoms.
5. Cavernous malformations: Abnormal collections of blood vessels in the brain that can cause a range of symptoms depending on their location and size.

Intracranial arterial diseases can be diagnosed using a variety of imaging tests, such as CT or MRI scans, ultrasound, and angiography. Treatment options may include medications to manage symptoms, surgery to repair or remove abnormal blood vessels, or endovascular procedures to treat conditions such as aneurysms and AVMs.

Prevention of intracranial arterial diseases includes managing risk factors such as high blood pressure, diabetes, and high cholesterol, as well as avoiding activities that increase the risk of head injury or trauma. Early diagnosis and treatment of these conditions can help to improve outcomes for patients with intracranial arterial diseases.

Some common types of psychomotor disorders include:

1. Dystonia: A movement disorder characterized by involuntary muscle contractions that can cause abnormal postures or movements.
2. Chorea: A condition marked by brief, irregular movements that can be writhing or jerky.
3. Athetosis: A slow, writhing movement that can affect the hands, face, and other parts of the body.
4. Tics: Sudden, repetitive movements or vocalizations that can be due to a variety of causes, such as Tourette's syndrome.
5. Parkinsonism: A group of disorders characterized by tremors, rigidity, bradykinesia (slowness of movement), and postural instability, often seen in conditions like Parkinson's disease or Huntington's disease.
6. Hemiballism: A condition where one side of the body is affected by involuntary movements, typically due to a stroke or other brain injury.
7. Gait abnormalities: Difficulty with walking or running due to problems with muscle coordination, balance, or other factors.
8. Oculomotor disorders: Abnormalities in eye movement, such as nystagmus (involuntary eye movements), can be a sign of a psychomotor disorder.
9. Stereotypic movements: Repetitive, purposeless movements that can occur in conditions like autism or other developmental disorders.
10. Hyperkinetic syndromes: Conditions characterized by excessive and/or purposeless movement, such as restless legs syndrome or tardive dyskinesia.

Psychomotor disorders can significantly impact an individual's quality of life, affecting their ability to perform daily tasks, communicate effectively, and maintain relationships. Treatment options vary depending on the specific condition but may include medication, physical therapy, occupational therapy, and behavioral interventions.

There are several potential causes of muscle hypertonia, including:

1. Neurological disorders such as cerebral palsy, Parkinson's disease, or multiple sclerosis
2. Musculoskeletal injuries or inflammation
3. Infections such as Lyme disease or viral infections
4. Metabolic disorders such as hypokalemia (low potassium levels) or hyperthyroidism
5. Adverse reactions to certain medications
6. Emotional stress or anxiety

Symptoms of muscle hypertonia can vary depending on the severity and location of the condition, but may include:

1. Stiffness and rigidity of the affected muscles
2. Pain or tenderness in the muscles
3. Limited range of motion in the affected joints
4. Fatigue or weakness in the affected limbs
5. Difficulty with movement and balance
6. Muscle spasms or cramping

Treatment for muscle hypertonia typically involves a combination of physical therapy, medication, and lifestyle modifications. Physical therapy may include stretching and strengthening exercises to improve range of motion and reduce stiffness, as well as techniques such as heat or cold therapy to relax the muscles. Medications such as muscle relaxants or anti-inflammatory drugs may be prescribed to reduce muscle spasms and inflammation. Lifestyle modifications such as regular exercise, proper nutrition, and stress management techniques can also help to reduce symptoms of muscle hypertonia. In severe cases, surgery may be necessary to release or lengthen the affected muscles.

Example sentence: The patient had a hemorrhage after the car accident and needed immediate medical attention.

The exact cause of hemangiomas is not known, but they are thought to be caused by an abnormal formation of blood vessels during fetal development. Hemangiomas are more common in infants and children, and they tend to grow rapidly during the first year of life. They are usually small and do not cause any symptoms, but can become larger and more complex over time.

The diagnosis of a hemangioma is based on a physical examination, imaging studies such as ultrasound or MRI, and a biopsy. Treatment for hemangiomas may include observation, steroid medications, or surgical removal if the lesion is causing symptoms or is large and unsightly.

The following are some of the key features of hemangioma, cavernous:

1. Location: Hemangiomas can occur anywhere in the body, but they are most common in the skin and subcutaneous tissue.
2. Composition: Hemangiomas are made up of abnormal and dilated blood vessels.
3. Size: Hemangiomas can range in size from a few millimeters to several centimeters in diameter.
4. Shape: Hemangiomas can be round or oval in shape, and may have a raised or depressed surface.
5. Color: Hemangiomas are typically red or purple in color, but can also be blue or brown.
6. Symptoms: Hemangiomas may cause symptoms such as pain, swelling, or bleeding, depending on their location and size.
7. Cause: The exact cause of hemangiomas is not known, but they are thought to be caused by an abnormal formation of blood vessels during fetal development.
8. Treatment: Treatment for hemangiomas may include observation, steroid medications, or surgical removal if the lesion is causing symptoms or is cosmetically unsightly.

The following are some of the key features of hemangioma, capillary:

1. Location: Hemangiomas can occur anywhere in the body, but they are most common in the skin and subcutaneous tissue.
2. Composition: Hemangiomas are made up of abnormal and dilated capillaries.
3. Size: Hemangiomas can range in size from a few millimeters to several centimeters in diameter.
4. Shape: Hemangiomas can be round or oval in shape, and may have a raised or depressed surface.
5. Color: Hemangiomas are typically red or purple in color, but can also be blue or brown.
6. Symptoms: Hemangiomas may cause symptoms such as pain, swelling, or bleeding, depending on their location and size.
7. Cause: The exact cause of hemangiomas is not known, but they are thought to be caused by an abnormal formation of capillaries during fetal development.
8. Treatment: Treatment for hemangiomas usually involves observation and monitoring, but may also include surgical removal or laser therapy in some cases.

It's important to note that while hemangiomas are not cancerous, they can be difficult to distinguish from other types of vascular lesions, and a biopsy may be necessary to confirm the diagnosis. If you suspect you have a hemangioma, it's important to consult with a qualified healthcare professional for an accurate diagnosis and appropriate treatment.

Hyponatremia can be caused by various factors, such as excessive fluid intake, certain medications, kidney or liver disease, and hormonal imbalances. Symptoms may include headache, nausea, vomiting, fatigue, muscle weakness, and in severe cases, seizures or coma.

Treatment for hyponatremia typically involves correcting the underlying cause of the condition. This may involve discontinuing certain medications, addressing any underlying medical conditions, or limiting fluid intake. In severe cases, hospitalization may be necessary to monitor and treat the condition. In some instances, sodium supplements or diuretics may be prescribed to help correct sodium levels.

It is important to note that hyponatremia can be a serious condition, and prompt medical attention should be sought if symptoms persist or worsen over time. A healthcare professional should be consulted for proper diagnosis and treatment.

Symptoms of heat stroke may include:

* High body temperature (usually above 104°F)
* Confusion or altered mental state
* Slurred speech
* Seizures or convulsions
* Dry, flushed skin with no sweating
* Rapid heartbeat
* Shallow breathing
* Nausea and vomiting

If you suspect someone has heat stroke, it is important to seek medical attention immediately. Treatment typically involves moving the person to a cooler location, removing excess clothing, and providing cool liquids to drink. In severe cases, hospitalization may be necessary to monitor and treat the condition.

Prevention is key in avoiding heat stroke, so it is important to take precautions during hot weather such as:

* Staying in air-conditioned spaces when possible
* Wearing lightweight, loose-fitting clothing
* Avoiding strenuous activity during the hottest part of the day (usually between 11am and 3pm)
* Drinking plenty of water to stay hydrated
* Taking regular breaks in shaded or cool areas
* Avoiding alcohol and caffeine, which can exacerbate dehydration.

By understanding the definition of heat stroke and taking preventative measures, you can help protect yourself and others from this potentially life-threatening condition.

Examples of brain diseases, metabolic, inborn include:

1. Phenylketonuria (PKU): A genetic disorder that affects the body's ability to break down the amino acid phenylalanine, leading to a buildup of toxic substances in the brain and blood.
2. Maple syrup urine disease (MSUD): Another genetic disorder that affects the body's ability to break down certain amino acids, resulting in a distinctive odor in the urine and potential brain damage if left untreated.
3. Mucopolysaccharidoses (MPS): A group of inherited diseases that affect the body's ability to produce or break down certain sugars, leading to progressive damage to various organs and systems, including the brain and nervous system.
4. Fabry disease: An inherited disorder caused by a deficiency of an enzyme called alpha-galactosidase A, which leads to the accumulation of a fatty substance in the body's cells and tissues, including the brain.
5. Mitochondrial disorders: A group of conditions caused by mutations or errors in the DNA of mitochondria, the energy-producing structures within cells. These disorders can affect various organs and systems, including the brain and nervous system.

These conditions are often treated with a combination of dietary restrictions, medication, and other therapies to manage symptoms and prevent complications. In some cases, bone marrow transplantation or enzyme replacement therapy may be necessary. Early detection and intervention can help improve outcomes for individuals with these conditions.

There are several types of ischemia, including:

1. Myocardial ischemia: Reduced blood flow to the heart muscle, which can lead to chest pain or a heart attack.
2. Cerebral ischemia: Reduced blood flow to the brain, which can lead to stroke or cognitive impairment.
3. Peripheral arterial ischemia: Reduced blood flow to the legs and arms.
4. Renal ischemia: Reduced blood flow to the kidneys.
5. Hepatic ischemia: Reduced blood flow to the liver.

Ischemia can be diagnosed through a variety of tests, including electrocardiograms (ECGs), stress tests, and imaging studies such as CT or MRI scans. Treatment for ischemia depends on the underlying cause and may include medications, lifestyle changes, or surgical interventions.

Eclampsia can occur at any time after the 20th week of pregnancy, but it is more common in the third trimester. It can also occur after delivery, especially in women who have a history of preeclampsia during pregnancy.

Symptoms of eclampsia can include:

1. Seizures or convulsions
2. Loss of consciousness or coma
3. Confusion or disorientation
4. Muscle weakness or paralysis
5. Vision problems or blurred vision
6. Numbness or tingling sensations in the hands and feet
7. Headaches or severe head pain
8. Abdominal pain or discomfort
9. Bladder or bowel incontinence
10. Rapid heart rate or irregular heartbeat.

Eclampsia is a medical emergency that requires immediate attention. Treatment typically involves delivery of the baby, either by cesarean section or vaginal birth, and management of the high blood pressure and any other complications that may have arisen. In some cases, medication may be given to help lower the blood pressure and prevent further seizures.

Preventive measures for eclampsia include regular prenatal care, careful monitoring of blood pressure during pregnancy, and early detection and treatment of preeclampsia. Women who have had preeclampsia in a previous pregnancy or who are at high risk for the condition may be advised to take aspirin or other medications to reduce their risk of developing eclampsia.

In summary, eclampsia is a serious medical condition that can occur during pregnancy and is characterized by seizures or coma caused by high blood pressure. It is a life-threatening complication of preeclampsia and requires immediate medical attention.

Migraine with aura is considered to be a more severe form of migraine than migraine without aura, which does not have the same neurological symptoms before the headache. Migraine with aura is also associated with a higher risk of other health problems, such as stroke and dementia.

There are several treatments available for migraine with aura, including medications that can help to reduce the frequency and severity of the headaches, as well as lifestyle changes such as avoiding triggers and getting regular exercise. It is important for people who experience migraine with aura to work closely with their healthcare provider to develop an effective treatment plan.

There are several different types of weight gain, including:

1. Clinical obesity: This is defined as a BMI of 30 or higher, and is typically associated with a range of serious health problems, such as heart disease, type 2 diabetes, and certain types of cancer.
2. Central obesity: This refers to excess fat around the waistline, which can increase the risk of health problems such as heart disease and type 2 diabetes.
3. Muscle gain: This occurs when an individual gains weight due to an increase in muscle mass, rather than fat. This type of weight gain is generally considered healthy and can improve overall fitness and athletic performance.
4. Fat gain: This occurs when an individual gains weight due to an increase in body fat, rather than muscle or bone density. Fat gain can increase the risk of health problems such as heart disease and type 2 diabetes.

Weight gain can be measured using a variety of methods, including:

1. Body mass index (BMI): This is a widely used measure of weight gain that compares an individual's weight to their height. A BMI of 18.5-24.9 is considered normal, while a BMI of 25-29.9 is considered overweight, and a BMI of 30 or higher is considered obese.
2. Waist circumference: This measures the distance around an individual's waistline and can be used to assess central obesity.
3. Skinfold measurements: These involve measuring the thickness of fat at specific points on the body, such as the abdomen or thighs.
4. Dual-energy X-ray absorptiometry (DXA): This is a non-invasive test that uses X-rays to measure bone density and body composition.
5. Bioelectrical impedance analysis (BIA): This is a non-invasive test that uses electrical impulses to measure body fat percentage and other physiological parameters.

Causes of weight gain:

1. Poor diet: Consuming high amounts of processed foods, sugar, and saturated fats can lead to weight gain.
2. Lack of physical activity: Engaging in regular exercise can help burn calories and maintain a healthy weight.
3. Genetics: An individual's genetic makeup can affect their metabolism and body composition, making them more prone to weight gain.
4. Hormonal imbalances: Imbalances in hormones such as insulin, thyroid, and cortisol can contribute to weight gain.
5. Medications: Certain medications, such as steroids and antidepressants, can cause weight gain as a side effect.
6. Sleep deprivation: Lack of sleep can disrupt hormones that regulate appetite and metabolism, leading to weight gain.
7. Stress: Chronic stress can lead to emotional eating and weight gain.
8. Age: Metabolism slows down with age, making it more difficult to maintain a healthy weight.
9. Medical conditions: Certain medical conditions such as hypothyroidism, Cushing's syndrome, and polycystic ovary syndrome (PCOS) can also contribute to weight gain.

Treatment options for obesity:

1. Lifestyle modifications: A combination of diet, exercise, and stress management techniques can help individuals achieve and maintain a healthy weight.
2. Medications: Prescription medications such as orlistat, phentermine-topiramate, and liraglutide can aid in weight loss.
3. Bariatric surgery: Surgical procedures such as gastric bypass surgery and sleeve gastrectomy can be effective for severe obesity.
4. Behavioral therapy: Cognitive-behavioral therapy (CBT) and other forms of counseling can help individuals develop healthy eating habits and improve their physical activity levels.
5. Meal replacement plans: Meal replacement plans such as Medifast can provide individuals with a structured diet that is high in protein, fiber, and vitamins, and low in calories and sugar.
6. Weight loss supplements: Supplements such as green tea extract, garcinia cambogia, and forskolin can help boost weight loss efforts.
7. Portion control: Using smaller plates and measuring cups can help individuals regulate their portion sizes and maintain a healthy weight.
8. Mindful eating: Paying attention to hunger and fullness cues, eating slowly, and savoring food can help individuals develop healthy eating habits.
9. Physical activity: Engaging in regular physical activity such as walking, running, swimming, or cycling can help individuals burn calories and maintain a healthy weight.

It's important to note that there is no one-size-fits-all approach to treating obesity, and the most effective treatment plan will depend on the individual's specific needs and circumstances. Consulting with a healthcare professional such as a registered dietitian or a physician can help individuals develop a personalized treatment plan that is safe and effective.

The following are some examples of consciousness disorders:

1. Coma: A coma is a state of deep unconsciousness in which an individual is unable to respond to any stimuli, including pain, light, or sound. This can be caused by traumatic brain injury, stroke, or metabolic disorders.
2. Vegetative State: A vegetative state is a condition in which an individual is awake but shows no signs of awareness or responsiveness. They may open their eyes, move their limbs, and respond to stimuli, but they do not exhibit any conscious thought or behavior.
3. Minimally Conscious State: A minimally conscious state is a condition in which an individual shows some signs of awareness and responsiveness, such as following simple commands or reacting to their environment. However, their cognitive functioning is severely impaired, and they may not be able to communicate effectively.
4. Locked-In Syndrome: Locked-in syndrome is a condition in which an individual is aware and awake but unable to move or communicate due to complete paralysis of all voluntary muscles except for the eyes. This can be caused by traumatic brain injury or a stroke.
5. Sleep Disorders: Sleep disorders such as narcolepsy, sleep apnea, and insomnia can also affect consciousness by altering the normal sleep-wake cycle.
6. Anoxic Brain Injury: Anoxic brain injury occurs when the brain is deprived of oxygen, leading to a loss of consciousness and potential long-term cognitive impairment.
7. Dementia: Dementia is a degenerative condition that affects cognitive functioning, including memory, language, and problem-solving abilities. Alzheimer's disease is the most common cause of dementia.
8. Brain Infections: Brain infections such as encephalitis and meningitis can cause alterations in consciousness by inflaming or damaging brain tissue.
9. Poisoning: Certain toxins, such as alcohol and drugs, can alter consciousness by disrupting normal brain functioning.
10. Electroconvulsive Therapy (ECT): ECT is a medical procedure that intentionally induces a seizure or loss of consciousness to treat severe mental illnesses such as depression.

It's important to note that the causes of altered consciousness can be diverse and complex, and a comprehensive diagnosis is often necessary to determine the underlying cause. Additionally, some forms of altered consciousness may be reversible with proper medical treatment, while others may result in long-term cognitive impairment or even death.

Symptoms of bacterial meningitis may include sudden onset of fever, headache, stiff neck, nausea, vomiting, and sensitivity to light. In severe cases, the infection can cause seizures, coma, and even death.

Bacterial meningitis can be diagnosed through a combination of physical examination, laboratory tests, and imaging studies such as CT or MRI scans. Treatment typically involves antibiotics to eradicate the infection, and supportive care to manage symptoms and prevent complications.

Early diagnosis and treatment are critical to prevent long-term damage and improve outcomes for patients with bacterial meningitis. The disease is more common in certain groups, such as infants, young children, and people with weakened immune systems, and it can be more severe in these populations.

Prevention of bacterial meningitis includes vaccination against the bacteria that most commonly cause the disease, good hand hygiene, and avoiding close contact with people who are sick.

Sources:

1. Dictionary of Medical Microbiology, Second Edition. Edited by A. S. Chakrabarti and S. K. Das. Springer, 2012.
2. Medical Microbiology, Fourth Edition. Edited by P. R. Murray, K. S. N air, and M. J. Laurence. Mosby, 2014.

Broca's aphasia is characterized by difficulty speaking in complete sentences, using correct grammar, and articulating words clearly. Individuals with Broca's aphasia may also experience difficulty understanding spoken language, although comprehension of written language may be relatively preserved.

Common symptoms of Broca's aphasia include:

1. Difficulty speaking in complete sentences or using correct grammar.
2. Slurred or slow speech.
3. Difficulty articulating words clearly.
4. Difficulty understanding spoken language.
5. Preservation of comprehension of written language.
6. Word-finding difficulties.
7. Difficulty with naming objects.
8. Difficulty with sentence construction.

Broca's aphasia is often caused by damage to the brain due to stroke, traumatic brain injury, or neurodegenerative diseases such as primary progressive aphasia. Treatment for Broca's aphasia typically involves speech and language therapy to improve communication skills and cognitive rehabilitation to improve language processing abilities.

Symptoms of venous thrombosis may include pain, swelling, warmth, and redness in the affected limb. In some cases, the clot can break loose and travel to the lungs, causing a potentially life-threatening condition called Pulmonary Embolism (PE).

Treatment for venous thrombosis typically involves anticoagulant medications to prevent the clot from growing and to prevent new clots from forming. In some cases, a filter may be placed in the vena cava, the large vein that carries blood from the lower body to the heart, to prevent clots from traveling to the lungs.

Prevention of venous thrombosis includes encouraging movement and exercise, avoiding long periods of immobility, and wearing compression stockings or sleeves to compress the veins and improve blood flow.

Cerebral Cortex. 15 (8): 1222-1233. doi:10.1093/cercor/bhi004. PMID 15616135. (Visual cortex). ... Ocular dominance columns were discovered in the 1960s by Hubel and Wiesel as part of their Nobel prize winning work on the ... Ocular dominance columns have since been found in many animals, such as ferrets, macaques, and humans. Notably, they are also ... Although the ocular dominance columns are formed before birth, there is a period after birth-formerly called a "critical period ...
From: BRI, CEREBRAL DOMINANCE, Conference Report #34. University of California, Los Angeles, 13-14. Połczyńska M, Japardi K, ... The development of human cerebral lateralization. In D. Frank Benson and E. Zaidel (Eds.) The Dual Brain. New York, NY: The ... Curtiss, Susan (1985). "The development of human cerebral lateralization". In Benson, D. Frank; Zaidel, E. (eds.). The Dual ... proving that her brain was not simply reversed in dominance for language. Curtiss and her team hypothesized that the critical ...
Foreign accents, language acquisition, and cerebral dominance revisited." Language Learning 20.2 (1970): 237-248. "On the ...
ISBN 978-0-88744-269-8. Hill, Jane (1970). "Foreign Accents, language acquisition, and cerebral dominance revisited". Language ... He reports observations of cerebral activation when reading and translating two languages. They found the most activated brain ... Other studies also highlight these problems: Stanislas Dehaene has investigated how cerebral circuits used to handling one ... ISBN 978-0-313-33100-8. Flege, James Emil; Mackay, Ian R. A.; Piske, Thorsten (2002). "Assessing bilingual dominance". Applied ...
Kimura, Doreen (1961). "Cerebral dominance and the perception of verbal stimuli". Canadian Journal of Psychology. 15 (3): 166- ...
Kimura, Doreen (1961). "Cerebral dominance and the perception of verbal stimuli". Canadian Journal of Psychology. 15 (3): 166- ...
Kimura D (1961). "Cerebral dominance and the perception of verbal stimuli". Canadian Journal of Psychology. 15 (3): 166-171. ... Kimura, Doreen (1961). "Cerebral dominance and the perception of verbal stimuli". Canadian Journal of Psychology. 15 (3): 166- ... He interpreted this result as indicating right-hemisphere dominance for pitch discrimination. During the early 1970s, Tim Rand ... Zatorre, Robert (1989). "Perceptual asymmetry on the dichotic fused words test and cerebral speech lateralization determined by ...
... and writing differ in relative cerebral dominance. ... if language is more related to the right hemisphere, then writing may be ...
Theodore H. Blau, The torque test: A measurement of cerebral dominance. 1974, American Psychological Association. Demarest, ... Measure Cerebral Dominance in Adults?". Perceptual and Motor Skills. 50 (1): 155-158. doi:10.2466/pms.1980.50.1.155. PMID ...
Holloway, RL; De (1982). "Some preliminary findings of the paleontology of cerebral dominance". American Journal of Physical ... LeMay, M (1976). "Morphological cerebral asymmetries of modern man, fossil man, and nonhuman primate". Annals of the New York ... Hopkins, WD; Marino, L (2000). "Asymmetries in cerebral width in nonhuman primate brains as revealed by magnetic resonance ... Phillips, Kimberly A.; Sherwood, Chet C. (June 11, 2007). "Cerebral Petalias and Their Relationship to Handedness in Capuchin ...
"Genomic and evolutionary analyses of asymmetrically expressed genes in human fetal left and right cerebral cortex". Cerebral ... Mixed-handedness or cross-dominance is the change of hand preference between different tasks. This is very uncommon in the ... However, the majority of left-handers have been found to have left-hemisphere language dominance-just like right-handers. Only ... A 2012 study showed that nearly 40% of children with cerebral palsy were left-handed, while another study demonstrated that ...
In humans, functional cerebral dominance is accompanied by subtle anatomical asymmetry. One function of the pineal gland is to ... Gross PM, Weindl A (December 1987). "Peering through the windows of the brain". Journal of Cerebral Blood Flow and Metabolism. ... Pineal tumors also can cause compression of the cerebral aqueduct, resulting in a noncommunicating hydrocephalus. Other ... supplied from the choroidal branches of the posterior cerebral artery. The pineal gland receives a sympathetic innervation from ...
He also carried out a program of studies on cerebral dominance. Among his collaborators were Fred Quadfasel, Jean Berko Gleason ...
Cerebral Cortex Cortical column Cerebral hemisphere ^ Ghosh A, Shatz CJ (March 1992). "Involvement of subplate neurons in the ... In the visual system thalamic axons to layer IV form ocular dominance columns and this segregation of thalamic axons is ... McConnell SK, Ghosh A, Shatz CJ (September 1989). "Subplate neurons pioneer the first axon pathway from the cerebral cortex". ... Subplate neurons (SPNs) are among the first generated neurons in the mammalian cerebral cortex [1]. These neurons disappear ...
Crow TJ, Close JP, Dagnall AM, Priddle TH (January 2009). "Where and what is the right shift factor or cerebral dominance gene ...
Intracarotid injection of Sodium Amytal for the lateralization of cerebral speech dominance. Experimental and clinical ...
Wada J (1949). "A new method for the determination of the side of cerebral speech dominance. A preliminary report of the intra- ... The Wada test, also known as the intracarotid sodium amobarbital procedure (ISAP), establishes cerebral language and memory ...
It is widely used as it is a non-invasive method of testing cerebral dominance. In a typical dichotic listening paradigm, the ...
Cerebral dominance or specialization has been studied in relation to a variety of human functions. With speech in particular, ... Ocular dominance: The eye preferred when binocular vision is not possible, as through a keyhole or monocular microscope. ... This has been called the "laterality-valence hypothesis". One sub-set of laterality in animals is limb dominance. Preferential ... Development and disorders of lateral dominance and the development of specialised centres and functions in the left and right ...
PMID 10452812 Finger S, Roe D. Gustave Dax and the early history of cerebral dominance. Arch Neurol. 1996 Aug;53(8):806-13. ... an overlooked chapter in the early history of cerebral dominance. J Hist Neurosci. 1996 Dec;5(3):228-40. PMID 11618743 Finger S ... The Temporal Lobe Theory and Other Contributions of an Overlooked Figure in the History of Language and Cerebral Dominance. ... On the contrary, he always claimed to be the first to espouse the theory of left hemisphere dominance for language and never ...
The Wada Test remains the gold standard for establishing cerebral dominance and is conducted worldwide prior to epilepsy ... including his description of the Wada test for cerebral hemispheric dominance of language function. ...
They are parasympathetic dominance, right-brain dominance. They often have a precipitous back of the head and prominent ... When fatigued, their cerebral reflexes become hyperactive. Their neck relaxes and the pubic bone begins to protrude. Their ... Taiheki type 1 has a strong tendency of cerebral sublimating of surplus energy into the intellectual activity. They turn their ... Taiheki type 2 has a habitual tendency to sublimation the diencephalon, hypersensitivity of the diencephalon, and cerebral ...
The mammalian cerebral cortex, the grey matter encapsulating the white matter, is composed of layers. The human cortex is ... Cortical minicolumn Ocular dominance column Predictive coding Radial unit hypothesis Mountcastle, Vernon (July 1957). "Modality ... The actual number of neurons within a single column is variable, and depends on the cerebral areas and thus the function of the ... A cortical column is a group of neurons forming a cylindrical structure through the cerebral cortex of the brain perpendicular ...
The corticospinal tract is a bundle of white matter which connects the cerebral cortex with motor neurons in the spinal cord. ... The presence of the RS+ gene promotes left-hemispheric dominance, in turn introducing a right-handedness bias which shifts the ... Carey, David P.; Johnstone, Leah T. (2014). "Quantifying cerebral asymmetries for language in dextrals and adextrals with ... "Cerebral Asymmetry and the Effects of Sex and Handedness on Brain Structure: A Voxel-Based Morphometric Analysis of 465 Normal ...
His work on cerebral dominance and reading disabilities was recognized by the International Reading Association in 1977 when he ...
Cerebral dominance theories (in the stutterer, no cerebral hemisphere takes the neurological lead) and theories of ... Developmental disabilities such as autism and neurological disorders such as cerebral palsy may also result in impaired ...
Crow, T J (1997). "Schizophrenia as failure of hemispheric dominance for language" (PDF). Trends in Neurosciences. Elsevier. 20 ... ISBN 978-0-521-80532-2. Grazi, E. (12 August 2013). "Cerebral ventricular size and cognitive impairment in... [Lancet. 1976] - ... Crow has also proven that people with Schizophrenia show less left-sided dominance for language. In the 1980s, Crow published ... a degree of enlargement of the cerebral ventricles) in individuals who have suffered from schizophrenia. Much subsequent work ...
Martindale, Hasenfus and Hines even suspected that the dominance of the right hemisphere contributed to the two basic elements ... They believed that Hitler's behavior was dominated by his right cerebral hemisphere, a situation that resulted in symptoms such ...
Most notably, Vaid's research in neuropsychology has clarified the role of the two cerebral hemispheres in bilingual language ... in contrast to the greater left hemisphere dominance for language among single language users. Recently she has examined ...
... including well-documented specializations in the brain's cerebral cortex and limbic systems, and the research into left-right ... Ned Herrmann described dominance of a particular thinking style with dominance with a portion of a brain hemisphere. The notion ... The Herrmann Brain Dominance Instrument (HBDI) is a system to measure and describe thinking preferences in people, developed by ... In his brain dominance model, Herrmann identifies four different modes of thinking: A. Analytical thinking Key words: logical, ...
... cerebral palsy or other conditions that affect muscle co-ordination and control. Athletes in class 31-34 compete in a seated ... marathon saw Paralympic champion El Amin Chentouf of Morocco finishing close to the Championship record to cement his dominance ...
Ueki, Y.; Mima, T.; Oga, T.; Ikeda, A.; Hitomi, T.; Fukuyama, H.; Nagamine, T.; Shibasaki, H. (February 2005). "Dominance of ... First, some researchers believe that this neurological disorder is due to abnormal communication between cerebral hemispheres. ... Movement disorders Chiari malformation Klippel-Feil Syndrome Dystonia Cerebral palsy Parkinson's disease Epilepsies Amyotrophic ...
We'll see how far this trend goes, but I suspect there will always be a place for a game which is totally cerebral in combat, ... Gasse, Nicholas (21 September 2010). "Xbox 360 dominance in Japan will come 'over time'". Play.tm. Ferrago Ltd. Archived from ... these systems have not shown as much market dominance in Eastern markets such as Japan, and only a few Western RPG titles have ...
Oxygenated blood is supplied by the anterior cerebral artery to most medial portions of the frontal lobes and to the anterior ... and the right hand will turn alien in the person with right-hemisphere dominance).[citation needed] In "the callosal variant", ... The patient had a ruptured aneurysm near the anterior cerebral artery, which resulted in the right hand being mirrored by the ... In the presence of unilateral damage to a single cerebral hemisphere, there is generally a gradual reduction in the frequency ...
Includes: Football 5-a-side - for visually impaired athletes Football 7-a-side - for athletes with cerebral palsy Amputee ... dominance of sports in the UK began to wane after the Factory Act of 1850, which significantly increased the recreation time ...
These signs suggestive of cerebral atrophy were associated with schizophrenia symptoms. In a follow up study, Silverton et al. ... Crow TJ (August 1997). "Schizophrenia as failure of hemispheric dominance for language". Trends Neurosci. 20 (8): 339-43. doi: ... and cerebral ventricular enlargement". Journal of Abnormal Psychology. 97. 97 (3): 496-498. doi:10.1037/0021-843X.97.4.496. ... and enlargement of brain ventricles suggestive of cerebral atrophy, Leigh Silverton and colleagues found that low birthweight ( ...
Hof, P. R.; Van der Gucht, E. (Jan 2007). "Structure of the cerebral cortex of the humpback whale, Megaptera novaeangliae ( ... and there is behavioral evidence for hemispheric dominance for vision.[citation needed] The evolution of encephalization in ... "Total number and volume of Von Economo neurons in the cerebral cortex of cetaceans". The Journal of Comparative Neurology. 515 ...
Mayers, Joshua (16 May 2011). "An unforgettable tifo Saturday: "Decades of Dominance". The Seattle Times. Retrieved 24 May 2011 ... for athletes with cerebral palsy. Parking the bus: when all the players on a team play defensively, usually when the team is ...
The marriage was brief: rendered comatose by a cerebral haemorrhage in her seventh month of pregnancy, Maria Isabel Patiño de ... giving a critique of the dominance of Neo-Liberalism in the governments of the First World. In its text he criticised their ...
Early recollections have also been found more accurate in their reflections of friendliness for men and dominance for women. ... There are reasons to believe that different associations within the cerebral hemisphere have an effect on remembering events ... Characteristics of early recollections are reflective of friendliness for males and dominance for females. Even when childhood ...
With the central hospital still under communist occupation, and no other medical service available she died of cerebral ... communist dictatorship and American dominance. Described by Daniel Ellsberg as an "outstanding Vietnamese journalist", he was a ...
Travers, Ben (19 October 2018). "Wanderlust Review: Toni Collette's Dominance of 2018 Continues in a Smart Netflix Series ... simply amazing in her professional debut as the girl with cerebral palsy." She joined the Sydney Theatre Company and, from ...
21: More Americans Are Getting the Updated Vaccine, BA.5 Continues Dominance in U.S." Frequent Business Traveller. Retrieved 21 ... specifically cerebral venous sinus thrombosis (CVST). On 20 March, the WHO and European Medicines Agency found no link to ...
According to Thurlow, the members of the "Populist" faction that challenged the ex-GBM faction's dominance in the late 1970s ... "the negro has a smaller brain and a much less complex cerebral structure" than whites; in the early 1980s, Nationalism Today ...
... reinforcing the dominance of the left hemisphere in language.[citation needed] The supramarginal gyrus is located just anterior ... is also known as Brodmann area 40 based on the brain map created by Korbinian Brodmann to define the structures in the cerebral ...
Cerebral hemorrhage, Subdural hematoma, Aneurysm, Hydrocephalus ("water on the brain"), Cerebral shunt, Meningioma (tumors), ... leading to the evolutionary success of widespread dominance of the human species across the planet. While the emphasis below is ... See also Cerebral infarction and Stroke recovery Disease theory of alcoholism - problem drinking is sometimes caused by a ... It has the same general structure as the brains of other mammals, but with a more developed cerebral cortex than any other, ...
She is cerebral and practical-it's hard to find practitioners who are both-and she is inclusive. She didn't just want to tell ... one of the little-noticed reasons for Mrs David's dominance of her audience in the 1950s was her miraculous sense of lucid ...
A mutation of the Nogo receptor in mice was found to extend the critical period for monocular dominance from around 20 - 32 ... The critical period hypothesis holds that first language acquisition must occur before cerebral lateralization completes, at ... For example, on the one hand, in kittens, a shift in ocular dominance resulting from monocular deprivation during the critical ... For example, PNN digestion by ABC chondroitinase in rats leads to a shift in ocular dominance upon monocular deprivation, which ...
With this technique she investigated how neurons in the cerebral cortex communicate with each other and found that connections ... ocular dominance plasticity in mouse visual cortex". Current Opinion in Neurobiology. Sensory systems. 16 (4): 451-459. doi: ...
... dominance, territoriality, and ritual displays. Paleomammalian complex This consists of the septum, amygdalae, hypothalamus, ... Neomammalian complex This consists of the cerebral neocortex, a structure found uniquely in higher mammals, and especially ...
... visual system has different critical periods for ocular dominance, visual acuity and binocular function as well as different ... skill that has been investigated using neuroimaging to date employs a network of brain regions spread across both cerebral ...
Autosomal dominance means affected parents have a 50% chance of passing on their mutated IRF6 allele to a their child. ... heart murmur and cerebral abnormalities, syndactyly of the hands, polythelia, ankyloglossia, and adhesions between the upper ...
The corpus callosum is the primary communication mechanism between the brain's two cerebral hemispheres. For example, ... It would make the appearance that there is a dual consciousness competing for dominance over the other for control of the brain ... ISBN 978-0-262-01401-4. Sperry, R (24 September 1982). "Some effects of disconnecting the cerebral hemispheres". Science. 217 ( ...
At UVA, Erisir's research has consisted of investigating ocular dominance column plasticity using several different techniques ... Cerebral Cortex. 29 (3): 921-936. doi:10.1093/cercor/bhx364. ISSN 1047-3211. Schecter, Rachel W.; Maher, Erin E.; Welsh, ...
The impact of Duchamp's Fountain changed the way people view art due to his focus upon "cerebral art" contrary to merely " ... As a feminist artist, Levine remakes works specifically by male artists who commandeered patriarchal dominance in art history ...
WBAY's cerebral palsy telethon both pre-dated and succeeded the national telethon for United Cerebral Palsy, which ran on ... but has maintained long-term dominance in the local ratings for most of its history. Until September 2012, when WFRV debuted ... The former gymnasium/auditorium is now called the WBAY Auditorium and is used as the studio for the station's cerebral palsy ... which has been broadcast on the station since 1954 and benefits Cerebral Palsy, Inc., a local organization involved in the care ...
Start Over You searched for: Subjects Dominance, Cerebral ✖Remove constraint Subjects: Dominance, Cerebral ...
... Author: Jacob, Heike; ... Cerebral integration of verbal and nonverbal emotional cues: Impact of individual nonverbal dominance. DSpace Repository. Login ...
Cerebral glucose metabolism was measured from all MRD patients twice, before and 3 months after rTMS, and from 20 healthy ... Dominance, Cerebral / physiology * Drug Resistance* * Energy Metabolism / physiology* * Female * Fluorodeoxyglucose F18 * ... mechanism of add-on repetitive transcranial magnetic stimulation in medication-resistant depression using cerebral glucose ...
Dominance, Cerebral (3). Attention (3). Social sciences (3). Connectionism (3). Perception (3) ...
Cerebral Dominance: Language Functions of The Left and Right Hemispheres[edit , edit source]. The perisylvian cortex of the ... Branch, C., Milner, B., Rasmussen, T. (1964). Intracarotid Sodium Amytal for the Lateralization of Cerebral Speech Dominance. ... Wada, J., Rasmussen, T. (1960). Intracarotid Injection of Sodium Amytal for the Lateralization of Cerebral Speech Dominance ... is a non-invasive method for examining event-related changes in cerebral blood flow velocity in the middle cerebral arteries( ...
Dominance, Cerebral / physiology* Actions. * Search in PubMed * Search in MeSH * Add to Search ...
Modifications of cerebral metabolism in rabbits in vivo under the influence of LSD-25 and cardiazol. Effects of certain ... Herold M Modifications du metabolisme cerebral chez le lapin in-vivo sous laction du LSD-25 et du cardiazol. Effets de ... This suggests the dominance of aerogenic glycolysis in spite of over-all diminution of oxidative processes (glutathione ... Cerebral circulation decreased. EEG: slow, high waves. . Methylandrostanolone: antagonizes the LSD effect on cerebral ...
Dominance of one cerebral hemisphere over the other in cerebral functions.. Terms. Dominance, Cerebral Preferred Term Term UI ... Dominance of one cerebral hemisphere over the other in cerebral functions.. Entry Term(s). Cerebral Dominance Hemispheric ... Cerebral Dominance Term UI T013122. Date03/29/1974. LexicalTag NON. ThesaurusID UNK (19XX). ... Dominance, Cerebral Preferred Concept UI. M0006749. Scope Note. ... Dominance, Cerebral. Tree Number(s). F02.830.297. G11.561.225. ...
Unfortunately, as we did not analyse brain tissues or cerebral spinal fluid, we cannot rule out higher toxin levels in these ... In Galicia, the phytoplankton successions, during the upwelling system dynamics, are characterized by the dominance of diatoms ... Xie, X.; Basile, M.; Mash, D.C. Cerebral uptake and protein incorporation of cyanobacterial toxin β-N-methylamino-L-alanine. ... Furthermore, dolphins with detectable levels of BMAA presented injuries in the cerebral cortex and increased β-amyloid plaques ...
Sasaki found regional cerebral blood flow reduction with frontal dominance and severe hypoperfusion around the corpus callosum. ... Cerebral perfusion pattern of idiopathic normal pressure hydrocephalus studied by SPECT and statistical brain mapping. Ann Nucl ... 38] However, Kahlon suggested that cine phase-contrast MRI measurements of stroke volume in the cerebral aqueduct are not ... Ultrasonography is not used for the diagnosis of NPH, although some researchers have suggested that reduced cerebral blood flow ...
From: BRI, CEREBRAL DOMINANCE, Conference Report #34. University of California, Los Angeles, 13-14. pdf1973 ... The development of human cerebral lateralization. In D. Frank Benson and E. Zaidel (Eds.) The Dual Brain. New York, NY: The ... Her work also demonstrates that the acquisition of grammar may be the essential trigger for normal cerebral organization of ...
Laboratories offers high tech male management devices for playful and/or serious cerebral power exchange and sensual dominance ...
Dominance cérébrale Entry term(s):. Cerebral Dominance. Dominances, Cerebral. Hemispheric Specialization. Specialization, ... Dominance, Cerebral - Preferred Concept UI. M0006749. Scope note. Dominance of one cerebral hemisphere over the other in ... Dominance of one cerebral hemisphere over the other in cerebral functions.. Allowable Qualifiers:. DE drug effects. GE genetics ... Dominance, Cerebral Entry term(s). Cerebral Dominance Dominances, Cerebral Hemispheric Specialization - Related but not broader ...
Interhemispheric relations and cerebral dominance. Baltimore, MD: John Hopkins Press. Tente links no:* Google ...
Dominance of one cerebral hemisphere over the other in cerebral functions.. Terms. Dominance, Cerebral Preferred Term Term UI ... Dominance of one cerebral hemisphere over the other in cerebral functions.. Entry Term(s). Cerebral Dominance Hemispheric ... Cerebral Dominance Term UI T013122. Date03/29/1974. LexicalTag NON. ThesaurusID UNK (19XX). ... Dominance, Cerebral Preferred Concept UI. M0006749. Scope Note. ... Dominance, Cerebral. Tree Number(s). F02.830.297. G11.561.225. ...
Cerebral neurobiological maturation processes. Proliferative phenomena, of routing. Regressive phenomena, of apoptosis ... Hemispheric lateralization and sexual dominance or differentiation. Sensitive-vulnerable periods. Brain plasticity ...
The high begins with a cerebral head rush following by an uplifting euphoric effect and a sense of focus.. Like all auto- ... Although Auto Gelato has a slight indica dominance, the high is uplifting and all in the head, meaning it wont leave you couch ... Effect: Cerebral, creative, long lasting, physical, powerful. Medical Conditions: Muscle spasms, chronic pain, inflammation, ...
Cerebral Dominance 37% * brain 28% * Language and developmental plasticity after perinatal stroke. Newport, E. L., Seydell- ...
Dominance, Cerebral 1 0 Note: The number of publications displayed in this table will differ from the number displayed in the ...
Cerebral dominance WL 337 Physiology of mental processes Back to top Neurologic Manifestations. ...
... crossed hand-eye dominance, and non-congruent hand-eye dominance compared to controls, but not the female patients. Cerebral ... In this study the possible relationships among handedness, eye dominance, and crossed and non congruent hand-eye dominance in ... Eye dominance was measured only by the near-far alignment test. Patients with schizophrenia had a significantly increased ... Dominância Cerebral/fisiologia , Lateralidade Funcional/fisiologia , Desempenho Psicomotor/fisiologia , Esquizofrenia/ ...
Master is more cerebral. He has the basic skills for maintaining a home properly, but no real desire to putter around tools. He ... Dominance, Dominance and submission, Life, Master, Slave, Submission, submissive , 2 Comments The moon shot: Sam Garcia D800E, ... Posted: October 7, 2014 , Author: dievca , Filed under: Master , Tags: BDSM, D/s, dievca, Dominance, Dominant, submissive , 22 ... Posted: June 3, 2015 , Author: dievca , Filed under: D/s Relationship, Master , Tags: BDSM, D/s, dievca, Dominance, Dominant, ...
I am extremely psychological and cerebral in my dominance. Because of my hyper intuition and driven focus to dig into my ...
I am extremely psychological and cerebral in my dominance. Because of my hyper intuition and driven focus to dig into my ...
I am extremely psychological and cerebral in my dominance. Because of my hyper intuition and driven focus to dig into my ...
COVID-19 Epidemiology during Delta Dominance Period Effects of School-Based Measures on COVID-19 COVID-19 Preventive Measures, ... Acute Cerebral Stroke with Multiple Infarctions and COVID-19, France, 2020. Emerging Infectious Diseases. 2020;26(9):2258-2260 ... Acute Cerebral Stroke with Multiple Infarctions and COVID-19, France, 2020 On This Page ... Cerebral magnetic resonance image (MRI) showing acute ischemic stroke in multiple vascular areas of 2 coronavirus disease ...
... but the resurgent dominance of the SPR-208 marksman rifle in Modern Warfare 2 shows that history is more prone to repetition ... Chalk it up to a cocktail of cerebral palsy and apathy, but Ive never been able to get a handle on sniping in multiplayer ... Infinity Ward would have us believe that history is written by the victors, but the resurgent dominance of the SPR-208 marksman ... Call of Duty faces an early balancing road bump as a single rifle asserts dominance across all game modes. ...
No article was found for Dominance, Cerebral and CREB1[original query]. File Formats Help:. How do I view different file ...
  • Very high resolution functional magnetic resonance imaging (fMRI) at a 4 Tesla (T) magnetic field was used to map ocular dominance regions in the human visual cortical layers using the blood oxygen level dependent (BOLD) contrast mechanism. (nih.gov)
  • The fMRI response from primary visual cortex (V1) exhibited a distribution of ocular dominance reminiscent of the single-cell recordings of Hubel and Wiesel. (nih.gov)
  • Although large vessel BOLD signals are easily detectable, regardless of field strength, they demonstrate a fMRI response to photic input that could not be used to distinguish ocular dominance. (nih.gov)
  • This necessitates the use of a difference paradigm to visualize the regions of ocular dominance accurately. (nih.gov)
  • This process is called ocular dominance. (nih.gov)
  • Hubel and Wiesel established that ocular dominance takes place within a limited window of early life called the critical period. (nih.gov)
  • New research on ocular dominance by Dr. Carla Shatz at Stanford, who was a graduate student with Hubel, has even pointed to a potential new drug target for restoring lost plasticity in Alzheimer's disease. (nih.gov)
  • The electric response evoked in the cerebral cortex by visual stimulation or stimulation of the visual pathways . (lookformedical.com)
  • Set of cell bodies and nerve fibers conducting impulses from the eyes to the cerebral cortex . (lookformedical.com)
  • of the cerebral cortex of the human brain. (nih.gov)
  • In addition to the use of magnetic resonance (MR) tech- the brain stem and the cerebral cortex. (nih.gov)
  • Various areas of the cerebral cortex, such as the sen- of the contrasted region is associated with the control or sory motor cortex, cingulate gyrus, and insula, have sensation of any physiological stimuli provided during the been found to become activated during volitional swal- acquisition of MR images. (nih.gov)
  • In collaboration with his Harvard colleague Torsten Wiesel, Hubel was among the first scientists to explore the inner workings of the cerebral cortex, the outermost layer of the brain that is responsible for higher functions like memory, planning, language, and making sense of the world around us. (nih.gov)
  • 12. Hemispheric language dominance studied with functional MR: preliminary study in healthy volunteers and patients with epilepsy. (nih.gov)
  • Thus a time course of be multifocal (5, 6, 16-20) and bilateral, with handed- gradient echo MRI images shows subtle changes in contrast ness-independent hemispheric dominance (16, 18, 19). (nih.gov)
  • There is still a lack of clarity about altered brain perfusion in NDPH and CM. This study aimed to investigate the cerebral perfusion variances of NDPH and CM using multi-delay pseudo-continuous arterial spin-labeled magnetic resonance imaging (pCASL-MRI). (biomedcentral.com)
  • Dominance of one cerebral hemisphere over the other in cerebral functions. (nih.gov)
  • Although Auto Gelato has a slight indica dominance, the high is uplifting and all in the head, meaning it won't leave you couch-locked. (sensibleseeds.com)
  • Auto Gelato is super-potent, with a cerebral uplifting high and is easy to grow and obtain excellent yields from. (dope-seeds.com)
  • 6. FMRI lateralization of expressive language in children with cerebral lesions. (nih.gov)
  • ings, using both block-trial and single-trial techniques, sug- gest that cerebral cortical regions activated during fMRI is a recently developed technique for functional study swallowing may not be specific to deglutitive function. (nih.gov)
  • Swallow-related cerebral cortical activity maps are lowing. (nih.gov)
  • Blood oxygenation level-dependent cerebral cortical mechanisms in both initiation and contrast is based on the fact that as blood is deoxygenated the regulation of volitional swallow. (nih.gov)
  • Direct investigation of diamagnetic oxyhemoglobin changes to paramagnetic deoxy- the human cerebral cortical areas involved in swallow- hemoglobin (3, 4). (nih.gov)
  • Cerebral magnetic resonance image (MRI) showing acute ischemic stroke in multiple vascular areas of 2 coronavirus disease patients, France. (cdc.gov)
  • The focus of the present study is detection of experimental evidence indicates an integral role for functional cerebral activity due to local changes in deoxyhe- moglobin concentration. (nih.gov)
  • Drawing much on the work of Molefi Kete Asante, chair of African Studies at Temple University, a distinct worldview is articulated which emphasizes the innate interconnected, animate, rhythmic, maternal essence of Africa, in contrast to an Eurocentric opposite of male dominance, positivist logic, and particulate conflict. (naturalgenesis.net)
  • All participants underwent 3D multi-delay pCASL-MRI to obtain cerebral perfusion data, including arrival-time-corrected cerebral blood flow (CBF) and arterial cerebral blood volume (aCBV). (biomedcentral.com)
  • Quantitative analysis of these signals revealed peak changes of +16 +/- 2% for HbO, -13 +/- 2% for HbR, +8 +/- 3% for total Hb, +83 +/- 9% for cerebral blood flow, and +1.4 +/- 0.1% for the BOLD MRI signal. (nih.gov)
  • The determination of various metabolites and electrolytes in carotid and v. jugularis blood of rabbits after 2 weeks of LSD treatment in comparison with controls yielded: decrease of the cerebral consumption of glucose and inorganic phosphate, diminished CO2 production, increased lactate consumption and increased glutathione reduction. (erowid.org)
  • Cerebral palsy (CP) is an umbrella term encompassing a group of non-progressive, non-contagious conditions that cause physical disability in human development. (blogspot.com)
  • The high begins with a cerebral head rush following by an uplifting euphoric effect and a sense of focus. (sensibleseeds.com)
  • These results suggest that levator muscle tone is influenced by eye dominance when ptosis is present. (nih.gov)
  • However, cerebral hemodynamic investigation was still rarely reported in CM, and there was no report of the cerebral hemodynamic features in NDPH. (biomedcentral.com)
  • Methylandrostanolone: antagonizes the LSD effect on cerebral metabolism, except the K loss. (erowid.org)
  • Viadril does not influence the LSD effect on cerebral circulation and consumption of glucose but it influences the effect on the consumption of phosphate and K, restoring these towards normal. (erowid.org)
  • The multi-delay pCASL technique can detect cerebral perfusion variation in patients with NDPH and CM. The cerebral perfusion changes may suggest different variations between NDPH and CM, which might provide hemodynamic evidence of these two types of primary headaches. (biomedcentral.com)
  • The presence of opposite yet reciprocal cerebral halves is a primary arbiter of our daily, public, and national lives. (naturalgenesis.net)
  • Check out the links below for some case studies specific to chiropractic care and cerebral palsy. (blogspot.com)

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