Desogestrel
Contraceptives, Oral, Synthetic
Contraceptives, Oral, Sequential
Norpregnenes
Contraceptives, Oral, Combined
Ethinyl Estradiol
Levonorgestrel
Spermatogenesis-Blocking Agents
Epitestosterone
Progesterone Congeners
Progestins
Contraceptives, Oral
Gonadotropins, Pituitary
Biotransformation
Department of health changes advice on third generation pills.(1/94)
(+info)A multicentre efficacy and safety study of the single contraceptive implant Implanon. Implanon Study Group. (2/94)
An open, multicentre study was performed to assess efficacy, safety and acceptability of the single-rod contraceptive implant Implanon. The study involved 635 young healthy women, who were sexually active and of childbearing potential. The women were followed up every 3 months over the entire study period. Originally the study was designed for 2 years, but was extended to 3 years in a group of 147 women from two centres. Altogether, 21 centres in nine different countries participated. The average age of the women was 29 years (range 18-42 years), of whom 83.5% had been pregnant in the past. No pregnancy occurred during treatment with Implanon, resulting in a Pearl Index of 0 (95% confidence interval: 0.0-0.2). In the first 2 years, 31% had discontinued the treatment. Of the 147 women in the study extension, nine discontinued (6%) treatment. Bleeding irregularities was the main reason for discontinuation during the first 2 years of use (17.2%) and adverse experiences in the third year (3.4%). Implant insertion and removal were fast and uncomplicated in the vast majority (97%) of cases. Return of fertility was prompt. In conclusion, Implanon has excellent contraceptive action during its lifetime of 3 years. The safety profile is acceptable and not essentially different from progestogens in general. (+info)A comparison of the inhibition of ovulation achieved by desogestrel 75 micrograms and levonorgestrel 30 micrograms daily. (3/94)
A randomized, double blind, group comparative study was performed over a 12 month period to compare inhibition of ovulation during the use of two (progestogen-only) oral contraceptives containing doses of 75 micrograms desogestrel or 30 micrograms levonorgestrel. Seventy-one female volunteers with regular cycles and established ovulation by ultrasonography and serum progesterone concentrations were recruited from an out-patient clinic in a university hospital and asked to participate in the study. Transvaginal ultrasonography and serum oestradiol, progesterone, luteinizing hormone (LH) and follicle stimulating hormone (FSH) measurements were performed throughout the 7th and 12th 28 day treatment period. Desogestrel at a dose of 75 micrograms showed a significant inhibition of ovulation compared to 30 micrograms levonorgestrel (P < 0.001). (+info)Venous thromboembolic disease and combined oral contraceptives: A re-analysis of the MediPlus database. (4/94)
In October 1995 the Committee on Safety of Medicines advised UK doctors and pharmacists that oral contraceptives containing desogestrel and gestodene were associated with double the risk of venous thromboembolic events (VTE) compared to pills containing other progestogens. In 1997 data was analysed from the MediPlus database of UK general practitioner records, which reported odds ratios for desogestrel and gestodene lower than that for levonorgestrel. Here the results of a more stringent nested case control analysis on the MediPlus database are reported. The study was larger and cases were verified. A crude incidence of idiopathic VTE was found amongst users of combined oral contraceptives of 4.6 per 10 000 exposed women years. Using levonorgestrel 150 microg + ethinyloestradiol 30 microg as reference, non-significant odds ratios of 1.1 (0.5-2.6) for desogestrel 150 microg + ethinyloestradiol 30 microg and 1.1 (0.5-2.4) for gestodene 75 microg + ethinyloestradiol 30 microg were found. The results of this study show no significant difference in risk between different formulations of combined oral contraceptive. (+info)The differential risk of oral contraceptives: the impact of full exposure history. (5/94)
Previous discussions have indicated that the small increases of risk of venous thromboembolism (VTE) associated with newer combined oral contraceptives (third generation, containing desogestrel and gestodene) may be attributed to bias due to cohort effects. In a case-control analysis, this may produce an overestimate of risk of newer preparations. In 10 centres in Germany and the UK, the Transnational Study analysed data from 502 women aged 16-44 years with VTE, and from 1864 controls matched for 5-year age group and region. Information on lifetime exposure history from all subjects was added to the dataset used in previous analyses and entered into a Cox regression model with time-dependent covariates. Based on 17 622 continuous exposure episodes comprising 47 914 person-years of observation, the adjusted hazard ratio (equivalent to odds ratio, OR) of VTE for the comparison of current users of third-generation versus current users of second-generation (primarily levonorgestrel compounds) combined oral contraceptives was 0.8 (0.5 to 1.3). The OR obtained in standard case-control analysis had been 1.5 (1.1 to 2.1). Adjustment for past exposures includes more information and appears more valid than the standard cross-sectional analysis. Using this approach, the Transnational Study data show no evidence for an increased risk of VTE with third- compared with second-generation combined oral contraceptives. (+info)The effect of etonogestrel on VEGF, oestrogen and progesterone receptor immunoreactivity and endothelial cell number in human endometrium. (6/94)
Contraceptive use often leads to disrupted endometrial bleeding patterns in women. In this study, two different contraceptive regimes (Mircette, a monophasic oral contraceptive and Implanon, a long-acting gestagen) were used and their effects on the immunoreactivity of vascular endothelial growth factor (VEGF), oestrogen receptor (ER), progesterone receptor (PR) and endothelial cell number were determined. During the untreated normal cycle, there was a significant increase (P = 0.005) in glandular VEGF immunoreactivity and a significant decrease (P < 0.05) in PR immunoreactivity in the mid- and late secretory phases compared with the proliferative phase. There was a significant positive correlation (gamma = 0.38, P = 0.046) between stromal VEGF immunoreactivity and endothelial cell number. This correlation was also apparent during treatment with Implanon, but not with Mircette. Disrupted bleeding patterns were associated with Implanon and, to a lesser extent, with Mircette. Both contraceptives significantly reduced glandular VEGF immunoreactivity. Implanon significantly increased (P = 0.016) glandular PR staining, but Mircette significantly reduced (P = 0.027) stromal PR staining when compared with secretory before-treatment biopsies. There were no changes in endothelial cell number or glandular or stromal ER during the normal cycle, or with use of either contraceptive. There was no association between the parameters measured with bleeding patterns and histological category. (+info)Bone mineral density during long-term use of the progestagen contraceptive implant Implanon compared to a non-hormonal method of contraception. (7/94)
An open, prospective, comparative study was done in healthy women, aged between 18 and 40 years, to study the effects of long-term etonogestrel treatment on bone mineral density (BMD). The control group used a non-hormone-medicated intrauterine device (IUD). The BMD was measured using a dual energy X-ray absorptiometry instrument. Measurements included the lumbar spine (L(2)-L(4)), the proximal femur (femoral neck, Ward's triangle, trochanter) and distal radius. The period of treatment was 2 years and 44 women in the Implanon group and 29 in the IUD group provided data. Groups were comparable at baseline with respect to age, weight, body mass index, BMD and 17beta-oestradiol status. Changes from baseline in BMD in the Implanon group were not essentially different from those in the IUD group. There was no relationship between 17beta-oestradiol concentrations and changes in BMD in this study population. The results of the present study indicate that Implanon((R)) can safely be used in young women who have not yet achieved their peak bone mass. (+info)Single monthly administration of the anti-progestagen Org 31710 in users of the 75 microg desogestrel progestagen-only pill: effects on pituitary-ovarian activity. (8/94)
Endocrine and ultrasound effects were studied of an intermittent (every 28 days) oral administration of 150 mg of the anti-progestagen Org 31710 during the continued daily use of 75 microg desogestrel (DSG) for progestagen-only contraception. A randomized, double-blind, placebo-controlled two-centre study was conducted in 50 healthy volunteers. Serum luteinizing hormone (LH), follicle stimulating hormone (FSH), oestradiol and progesterone concentrations, and follicle number and size were studied, as well as endometrial thickness, which was assessed by transvaginal sonography at least twice weekly during a single medication cycle (cycle 3-5). Forty-eight women were evaluated (Org 31710, n = 25; placebo, n = 23). Seven ovulations were observed in the treated group versus none in the placebo group. LH concentrations were higher on days 9 and 11 and oestradiol concentrations lower on day 3 in the treated group, irrespective of whether ovulation occurred. No parameter could predict ovulation. Endometrial thickness was greater on cycle days 7-13 and 19 in the treated group. However, within the Org 31710 group, no significant differences were found in volunteers who did or did not ovulate. Observed differences may be attributed to a competitive effect of Org 31710 with progestagen-induced suppression of the pituitary-ovarian axis, altered oestradiol feedback mechanisms, and/or altered receptor availability. (+info)Desogestrel is a synthetic form of progestin, which is a female sex hormone. It is used in various forms of hormonal contraception such as birth control pills, patches, and vaginal rings to prevent pregnancy. Desogestrel works by preventing ovulation (the release of an egg from the ovaries), thickening cervical mucus to make it harder for sperm to reach the egg, and thinning the lining of the uterus to make it less likely for a fertilized egg to implant.
Desogestrel is also used in some hormone replacement therapies (HRT) to treat symptoms of menopause such as hot flashes and vaginal dryness. It may be prescribed alone or in combination with estrogen.
Like all hormonal contraceptives, desogestrel has potential side effects, including irregular menstrual bleeding, headaches, mood changes, breast tenderness, and nausea. In rare cases, it may also increase the risk of blood clots, stroke, or heart attack. It is important to discuss the risks and benefits of desogestrel with a healthcare provider before using it.
Oral contraceptives, also known as "birth control pills," are synthetic hormonal medications that are taken by mouth to prevent pregnancy. They typically contain a combination of synthetic versions of the female hormones estrogen and progesterone, which work together to inhibit ovulation (the release of an egg from the ovaries), thicken cervical mucus (making it harder for sperm to reach the egg), and thin the lining of the uterus (making it less likely that a fertilized egg will implant).
There are several different types of oral contraceptives, including combination pills, progestin-only pills, and extended-cycle pills. Combination pills contain both estrogen and progestin, while progestin-only pills contain only progestin. Extended-cycle pills are a type of combination pill that are taken for 12 weeks followed by one week of placebo pills, which can help reduce the frequency of menstrual periods.
It's important to note that oral contraceptives do not protect against sexually transmitted infections (STIs), so it's still important to use barrier methods like condoms if you are at risk for STIs. Additionally, oral contraceptives can have side effects and may not be suitable for everyone, so it's important to talk to your healthcare provider about the potential risks and benefits before starting to take them.
Oral contraceptives, sequential, are a type of birth control medication that involves taking two different hormonal preparations in a specific sequence to mimic the natural menstrual cycle. The first hormone preparation contains estrogen and is taken for 16-21 days, followed by a second hormone preparation containing both estrogen and progestin for 7 days. This regimen causes the lining of the uterus to thin, making it less likely for a fertilized egg to implant, and also thickens cervical mucus, which can prevent sperm from reaching the egg. Sequential oral contraceptives are not commonly used in the United States due to their higher risk of side effects compared to other forms of oral contraceptives.
Norpregnenes are a class of steroids that are produced by the metabolism of progesterone and other pregnanes. They are characterized by the absence of a double bond between carbons 4 and 5, and the presence of a ketone group at carbon 3. Some examples of norpregnenes include dehydroepiandrosterone (DHEA), androstenedione, and pregnenolone. These steroids are important intermediates in the biosynthesis of various hormones, including cortisol, aldosterone, androgens, and estrogens. They play a role in various physiological processes such as sexual development, immune function, and stress response.
Oral combined contraceptives, also known as "the pill," are a type of hormonal birth control that contain a combination of synthetic estrogen and progestin. These hormones work together to prevent ovulation (the release of an egg from the ovaries), thicken cervical mucus to make it harder for sperm to reach the egg, and thin the lining of the uterus to make it less likely for a fertilized egg to implant.
Combined oral contraceptives come in various brands and forms, such as monophasic, biphasic, and triphasic pills. Monophasic pills contain the same amount of hormones in each active pill, while biphasic and triphasic pills have varying amounts of hormones in different phases of the cycle.
It is important to note that oral combined contraceptives do not protect against sexually transmitted infections (STIs) and should be used in conjunction with condoms for safer sex practices. Additionally, there are potential risks and side effects associated with oral combined contraceptives, including an increased risk of blood clots, stroke, and heart attack, especially in women who smoke or have certain medical conditions. It is essential to consult a healthcare provider before starting any hormonal birth control method to determine if it is safe and appropriate for individual use.
Ethinyl estradiol is a synthetic form of the hormone estrogen that is often used in various forms of hormonal contraception, such as birth control pills. It works by preventing ovulation and thickening cervical mucus to make it more difficult for sperm to reach the egg. Ethinyl estradiol may also be used in combination with other hormones to treat menopausal symptoms or hormonal disorders.
It is important to note that while ethinyl estradiol can be an effective form of hormonal therapy, it can also carry risks and side effects, such as an increased risk of blood clots, stroke, and breast cancer. As with any medication, it should only be used under the guidance and supervision of a healthcare provider.
Levonorgestrel is a synthetic form of the natural hormone progesterone, which is used in various forms of birth control and emergency contraceptives. It works by preventing ovulation (the release of an egg from the ovaries), thickening cervical mucus to make it harder for sperm to reach the egg, and thinning the lining of the uterus to make it less likely for a fertilized egg to implant.
Medically, Levonorgestrel is classified as a progestin and is available in various forms, including oral tablets, intrauterine devices (IUDs), and emergency contraceptive pills. It may also be used to treat endometriosis, irregular menstrual cycles, and heavy menstrual bleeding.
It's important to note that while Levonorgestrel is a highly effective form of birth control when used correctly, it does not protect against sexually transmitted infections (STIs). Therefore, condoms should still be used during sexual activity if there is any risk of STI transmission.
Spermatogenesis-blocking agents are a class of medications or substances that inhibit or block the process of spermatogenesis, which is the production of sperm in the testicles. These agents can work at various stages of spermatogenesis, including reducing the number of spermatozoa (sperm cells) or preventing the formation of mature sperm.
Examples of spermatogenesis-blocking agents include:
1. Hormonal agents: Certain hormones or hormone-like substances can interfere with the production of sperm. For example, analogs of gonadotropin-releasing hormone (GnRH) and antiandrogens can suppress the release of testosterone and other hormones necessary for spermatogenesis.
2. Alkylating agents: These are chemotherapy drugs that can damage DNA and prevent the division and multiplication of cells, including sperm cells. Examples include cyclophosphamide and busulfan.
3. Other chemicals: Certain industrial chemicals, such as ethylene glycol ethers and dibromochloropropane (DBCP), have been shown to have spermatogenesis-blocking properties.
4. Radiation therapy: High doses of radiation can also damage the testicles and inhibit sperm production.
It's important to note that spermatogenesis-blocking agents are often used for medical purposes, such as treating cancer or preventing pregnancy, but they can have significant side effects and should only be used under the guidance of a healthcare professional.
Epitestosterone is a steroid hormone that is structurally similar to testosterone. It is produced in the body, primarily in the testes and adrenal glands, and is a natural component of human urine. Epitestosterone is a weak androgen, meaning it has minimal male sex hormone effects.
The ratio of epitestosterone to testosterone (T/E ratio) in urine is often used as a marker for the detection of doping with anabolic steroids, which are synthetic versions of testosterone. In athletes who have not taken performance-enhancing drugs, the T/E ratio is typically less than 1. However, when anabolic steroids are used, the level of testosterone in the body increases, while the level of epitestosterone remains relatively unchanged, leading to a higher T/E ratio.
Medical professionals and anti-doping agencies use a specific cutoff value for the T/E ratio to determine if an individual has violated doping regulations. It's important to note that some individuals may have naturally higher T/E ratios due to genetic factors, which can complicate the interpretation of test results in anti-doping tests.
Progesterone congeners refer to synthetic or naturally occurring compounds that are structurally similar to progesterone, a steroid hormone involved in the menstrual cycle, pregnancy, and embryogenesis. These compounds have similar chemical structures to progesterone and may exhibit similar physiological activities, although they can also have unique properties and uses. Examples of progesterone congeners include various synthetic progestins used in hormonal contraceptives and other medical treatments.
Progestins are a class of steroid hormones that are similar to progesterone, a natural hormone produced by the ovaries during the menstrual cycle and pregnancy. They are often used in hormonal contraceptives, such as birth control pills, shots, and implants, to prevent ovulation and thicken the cervical mucus, making it more difficult for sperm to reach the egg. Progestins are also used in menopausal hormone therapy to alleviate symptoms of menopause, such as hot flashes and vaginal dryness. Additionally, progestins may be used to treat endometriosis, uterine fibroids, and breast cancer. Different types of progestins have varying properties and may be more suitable for certain indications or have different side effect profiles.
Oral contraceptives, also known as "birth control pills," are medications taken by mouth to prevent pregnancy. They contain synthetic hormones that mimic the effects of natural hormones estrogen and progesterone in a woman's body, thereby preventing ovulation, fertilization, or implantation of a fertilized egg in the uterus.
There are two main types of oral contraceptives: combined pills, which contain both estrogen and progestin, and mini-pills, which contain only progestin. Combined pills work by preventing ovulation, thickening cervical mucus to make it harder for sperm to reach the egg, and thinning the lining of the uterus to make it less likely for a fertilized egg to implant. Mini-pills work mainly by thickening cervical mucus and changing the lining of the uterus.
Oral contraceptives are highly effective when used correctly, but they do not protect against sexually transmitted infections (STIs). It is important to use them consistently and as directed by a healthcare provider. Side effects may include nausea, breast tenderness, headaches, mood changes, and irregular menstrual bleeding. In rare cases, oral contraceptives may increase the risk of serious health problems such as blood clots, stroke, or liver tumors. However, for most women, the benefits of using oral contraceptives outweigh the risks.
Gonadotropins are hormones produced and released by the anterior pituitary gland, a small endocrine gland located at the base of the brain. These hormones play crucial roles in regulating reproduction and sexual development. There are two main types of gonadotropins:
1. Follicle-Stimulating Hormone (FSH): FSH is essential for the growth and development of follicles in the ovaries (in females) or sperm production in the testes (in males). In females, FSH stimulates the maturation of eggs within the follicles.
2. Luteinizing Hormone (LH): LH triggers ovulation in females, causing the release of a mature egg from the dominant follicle. In males, LH stimulates the production and secretion of testosterone in the testes.
Together, FSH and LH work synergistically to regulate various aspects of reproductive function and sexual development. Their secretion is controlled by the hypothalamus, which releases gonadotropin-releasing hormone (GnRH) to stimulate the production and release of FSH and LH from the anterior pituitary gland.
Abnormal levels of gonadotropins can lead to various reproductive disorders, such as infertility or menstrual irregularities in females and issues related to sexual development or function in both sexes. In some cases, synthetic forms of gonadotropins may be used clinically to treat these conditions or for assisted reproductive technologies (ART).
Sperm count, also known as sperm concentration, is the number of sperm present in a given volume of semen. The World Health Organization (WHO) previously defined a normal sperm count as at least 20 million sperm per milliliter of semen. However, more recent studies suggest that fertility may be affected even when sperm counts are slightly lower than this threshold. It's important to note that sperm count is just one factor among many that can influence male fertility. Other factors, such as sperm motility (the ability of sperm to move properly) and morphology (the shape of the sperm), also play crucial roles in successful conception.
Biotransformation is the metabolic modification of a chemical compound, typically a xenobiotic (a foreign chemical substance found within an living organism), by a biological system. This process often involves enzymatic conversion of the parent compound to one or more metabolites, which may be more or less active, toxic, or mutagenic than the original substance.
In the context of pharmacology and toxicology, biotransformation is an important aspect of drug metabolism and elimination from the body. The liver is the primary site of biotransformation, but other organs such as the kidneys, lungs, and gastrointestinal tract can also play a role.
Biotransformation can occur in two phases: phase I reactions involve functionalization of the parent compound through oxidation, reduction, or hydrolysis, while phase II reactions involve conjugation of the metabolite with endogenous molecules such as glucuronic acid, sulfate, or acetate to increase its water solubility and facilitate excretion.
Desogestrel
Progestogen (medication)
Gestodene
Birth control pill formulations
List of combined sex-hormonal preparations
Combined oral contraceptive pill
Estradiol cyclooctyl acetate
Ethinylestradiol
Levonorgestrel
Etonogestrel
Acetomepregenol
Anagestone acetate
Nandrolone
Progestogen-only pill
Norethisterone
Birth control
Drospirenone
Feminizing hormone therapy
HAIR-AN syndrome
Allylestrenol
Estrogen (medication)
Norsteroid
Pharmacodynamics of progesterone
Venous thrombosis
Azalia
List of progestogens available in the United States
Estradiol-containing birth control pill
Hana
Contraceptive rights in New Zealand
Extended cycle combined hormonal contraceptive
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Ethinylestradiol5
- Desogestrel is available alone in the form of 75 μg oral tablets and at a dose of 150 μg in combination with 20 or 30 μg ethinylestradiol in oral tablets. (wikipedia.org)
- Because it contains both ethinylestradiol and desogestrel as active components, this drug is categorized as a hormonal combination agent. (canadapharmacy-usa.com)
- Specifically, it contains 30 mcg of ethinylestradiol and 150 mcg of desogestrel. (canadapharmacy-usa.com)
- This medication is a hormonal combined agent as it consists of two active ingredients: 30mcg of ethinylestradiol and 150mcg of desogestrel. (4rx.net)
- Ethinylestradiol + Desogestrel (Possibly safe. (e-lactancia.org)
Ethinyl estradiol and ethinyl estradiol2
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Desogen2
- Desogen contains a combination of of female hormones desogestrel and ethinyl estradiol, they prevent pregnacy. (blueorangepharma.cc)
- Desogen (desogestrel and ethinyl estradiol) Tablets are indicated for the prevention of pregnancy in women who elect to use this product as a method of contraception. (ncats.io)
Etonogestrel9
- Inducers of liver enzymes can increase the metabolism of desogestrel and etonogestrel and reduce their circulating levels. (wikipedia.org)
- CYP3A4 inhibitors including strong inhibitors like clarithromycin, itraconazole, and ketoconazole and moderate inhibitors like diltiazem, erythromycin, and fluconazole may increase levels of desogestrel and etonogestrel. (wikipedia.org)
- Receptor binding studies, as well as studies in animals, have shown that etonogestrel, the biologically active metabolite of desogestrel, combines high progestational activity with minimal intrinsic androgenicity (91,92). (nih.gov)
- Desogestrel is rapidly and almost completely absorbed and converted into etonogestrel, its biologically active metabolite. (nih.gov)
- Desogestrel is converted in the body into the active metabolite etonogestrel. (janusinfo.se)
- A comparison of desogestrel and etonogestrel. (janusinfo.se)
- Based on this, it can be assumed that there is a risk associated with the use of etonogestrel/desogestrel and that these substances appear additive with other androgenic gestagens in the environment. (janusinfo.se)
- Therefore, there is no clear environmental reason to choose etonogestrel before desogestrel or vice versa. (janusinfo.se)
- Desogestrel is a prodrug of etonogestrel (3-keto-desogestrel) which was approved as oral contraceptove medicine. (ncats.io)
Ethinyloestradiol or desogestrel1
- You should not use Novelon® if any of the following apply to you: you have an allergy to ethinyloestradiol or desogestrel, or to any of the other substances found in Novelon® contraceptive tablets. (canadapharmacy-usa.com)
Marvelon1
- Marvelon actually contains desogestrel. (hdc.org.nz)
Tablets5
- SIMLIYA TM also contains 5 light blue round tablets containing 0.01 mg ethinyl estradiol USP (19-nor-17 alpha-pregna-1,3,5 (10)-trien-20-yne--3,17-diol) and inactive ingredients which include colloidal silicon dioxide, FD&C blue no. 1, lactose monohydrate, povidone, pregelatinized starch (maize), stearic acid and vitamin E. The molecular weights for desogestrel and ethinyl estradiol are 310.48 and 296.40 respectively. (nih.gov)
- For this reason, combination oral contraceptives, including Enskyce™ (desogestrel and ethinyl estradiol tablets USP), should not be used by women who are over 35 years of age and smoke. (nih.gov)
- Enskyce™ (desogestrel and ethinyl estradiol tablets USP) provides an oral contraceptive regimen of 21 light orange round tablets each containing 0.15 mg desogestrel (13-ethyl-11-methylene-18,19-dinor-17 alpha-pregn-4-en-20-yn-17-ol) and 0.03 mg ethinyl estradiol (19-nor-17 alpha-pregna-1,3,5 (10)-trien-20-yne-3,17,diol). (nih.gov)
- be an old tactic, but it still works today.As you are organizing your items for the sale, label each (desogestrel tablets bp cerazette in hindi). (rebirthgames.com)
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Cerazette1
- Fass environmental information for Cerazette (desogestrel) from MSD (downloaded 2021-07-06). (janusinfo.se)
Gestodene1
- It is described as a third generation oral contraceptive, containing the progestogens desogestrel or gestodene. (hdc.org.nz)
Pill5
- The Novelon® contraceptive pill contains a mixture of two hormones, ethinyloestradiol, which is a synthetic oestrogen, and desogestrel, which is a synthetic progestogen. (canadapharmacy-usa.com)
- Desogestrel ethinyl estradiol is a type of combination birth control pill. (rxguide.org)
- The growing number desogestrel sandoz azalia of state laws that allow marijuana for medical use contributes to teen perceptions that marijuana is not a harmful drug, said Dr. The blood supply increases, the network of veins become more apparent, all in preparation mini pill desogestrel weight loss for the onslaught of mothers liquid gold. (rebirthgames.com)
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Venous thromboembolism2
- Serious side effects of combined oral contraceptives containing desogestrel may include venous thromboembolism, arterial thromboembolism, hormone-dependent tumors (e.g., liver tumors, breast cancer), and melasma. (wikipedia.org)
- The number of extra cases of venous thromboembolism per year per 10 000 treated women was lowest for levonorgestrel (6, 95% confidence interval 5 to 7) and norgestimate (6, 5 to 8), and highest for desogestrel (14, 11 to 17) and cyproterone (14, 11 to 17). (bmj.com)
Vaginal1
- Contraindications of desogestrel include: Allergy to desogestrel or any other ingredients Active thrombosis (deep vein thrombosis or pulmonary embolism) Jaundice or severe liver disease Hormone-sensitive cancers (e.g., breast cancer) Unexplained vaginal bleeding Desogestrel is not indicated for use in pregnancy. (wikipedia.org)
Include nausea1
- The most common side effects of desogestrel ethinyl estradiol include nausea, vomiting, headache, breast tenderness, and spotting between periods. (rxguide.org)
Progestogens3
- Desogestrel is a progestin, or a synthetic progestogen, and hence is an agonist of the progesterone receptor, the biological target of progestogens like progesterone. (wikipedia.org)
- Several synthetic progestogens contain androgenic activity, however the active ingredient in Novelon®, desogestrel, has extremely low levels of androgenic activity. (canadapharmacy-usa.com)
- Some synthetic progestogens have androgenic activity, but desogestrel in Novelon®, has very weak androgenic activity, which has a beneficial effect on androgen-related skin disorders such as acne and hirsutism (unwanted hair). (4rx.net)
Synthetic progestogen1
- Novelon® contraceptive pills contain a combination of two hormones, ethinyloestradiol, a synthetic oestrogen, and desogestrel, a synthetic progestogen, which work together to protect against unwanted pregnancy. (4rx.net)
Kaufen2
- This is when we first started the All-Mountain Terrain Park: desogestrel aristo online kaufen ohne rezept. (rebirthgames.com)
- is pretty desogestrel pille online kaufen healthy with very little red meat, but chicken and fish, lots of veg and fruit So a reasonable. (rebirthgames.com)
Hormones3
- Desogestrel ethinyl estradiol is a combination of two hormones, desogestrel and ethinyl estradiol, used as a contraceptive to prevent pregnancy. (rxguide.org)
- It is a combination of two hormones, desogestrel and ethinyl estradiol, and is available in both oral and transdermal forms. (rxguide.org)
- Both of these drugs contain the same strengths of two female hormones: ethinyl estradiol (an estrogen) and desogestrel (a progestin). (healthline.com)
Pille2
- and Resources Protection Committee of NPC to the Central Committee and the pille desogestrel aristo keine periode State Council on the development. (rebirthgames.com)
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Progesterone2
- Desogestrel acts selectively binding to progesterone receptor and enchancing its activity. (ncats.io)
- However, with synthetic progesterone, desogestrel in case of Aizea, the body will mistake the active ingredient for naturally produced progesterone, causing it to "think" that the ovulation already took place - as indicated by perceived increase in the levels of progesterone. (drseb.com)
Enskyce1
- In the third cycle of use after a single dose of Enskyce, maximum concentrations of 3-keto-desogestrel of 2,805 ± 1,203 pg/mL (mean ± SD) are reached at 1.4 ± 0.8 hours. (nih.gov)
Progestin2
- Desogestrel is a progestin medication which is used in birth control pills for women. (wikipedia.org)
- Desogestrel is sometimes referred to as a "third-generation" progestin. (wikipedia.org)
Pregnancy2
- Desogestrel ethinyl estradiol is more than 99% effective at preventing pregnancy when taken as directed. (rxguide.org)
- Desogestrel Ethinyl Estradiol is used for the prevention of pregnancy in women who choose to use an oral contraceptive. (rxguide.org)
Prescriptions1
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Medication2
- Yes, desogestrel ethinyl estradiol is a prescription medication. (rxguide.org)
- The dosage and administration of Desogestrel Ethinyl Estradiol depends on the form of the medication being used. (rxguide.org)
Prescription4
- You will need to talk to your doctor to get a prescription for desogestrel ethinyl estradiol. (rxguide.org)
- May desogestrel-ethinyl estradiol prescription you please extend them a little from next time? (rebirthgames.com)
- This pilule desogestrel mylan oubli includes prescription and over-the-counter medicines, and dietary supplements such as vitamins and herbs. (rebirthgames.com)
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Products1
- Concerns about the Ortho Evra transdermal patch and desogestrel-containing products also are discussed. (uspharmacist.com)
Body1
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Birth1
- Desogestrel is used in hormonal contraception in women, specifically in birth control pills. (wikipedia.org)
Levels2
- The minimum plasma levels of 3-keto-desogestrel at steady state are 1,400 ± 560 pg/mL. (nih.gov)
- This indicates that the kinetics of 3-keto-desogestrel are non-linear due to an increase in binding of 3-keto-desogestrel to sex hormone-binding globulin in the cycle, attributed to increased sex hormone-binding globulin levels which are induced by the daily administration of ethinyl estradiol. (nih.gov)
Side effects2
- In safety studies, dosages of up to 750 μg/day desogestrel in women showed no adverse effects on laboratory and various other parameters and produced no reported subjective side effects. (wikipedia.org)
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Risk2
- The use of desogestrel (sales data Sweden 2018) has been considered to result in moderate environmental risk. (janusinfo.se)
- Det europeiske legemiddelkontorets komité for legemiddelovervåking - PRAC (Pharmacovigilance Risk Assessment Committe), utarbeider en rekke anbefalinger til oppdatering av produktinformasjon og regulatoriske tiltak som følge av signalutredning. (legemiddelverket.no)
Combination1
- In addition to hormonal contraception, desogestrel has been used in combination with estrogens such as estradiol as a component of menopausal hormone therapy. (wikipedia.org)
High2
- Desogestrel can also cause changes in total, LDLTooltip low-density lipoprotein, and HDLTooltip high-density lipoprotein cholesterol. (wikipedia.org)
- Desogestrel has very high chronic toxicity. (janusinfo.se)
Single1
- The mean AUC 0-∞ for 3-keto-desogestrel at single dose is significantly lower than the mean AUC 0-24 at steady state. (nih.gov)
Approximately1
- The elimination half-life for 3-keto-desogestrel is approximately 38 ± 20 hours at steady state. (nih.gov)
Human1
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Addition1
- In addition to 3-keto-desogestrel, other phase I metabolites are 3α-OH-desogestrel, 3β-OH-desogestrel, and 3α-OH-5α-H-desogestrel. (nih.gov)