Hearing Loss, Sensorineural
Evoked Potentials, Auditory, Brain Stem
Hair Cells, Auditory
Hearing Loss, Central
Organ of Corti
Hair Cells, Auditory, Inner
Jervell-Lange Nielsen Syndrome
Transcription Factor Brn-3C
KCNQ Potassium Channels
Persons With Hearing Impairments
Hearing Loss, Conductive
Hair Cells, Auditory, Outer
Bulbar Palsy, Progressive
National Institute on Deafness and Other Communication Disorders (U.S.)
Variations in genetic assessment and recurrence risks quoted for childhood deafness: a survey of clinical geneticists. (1/1607)We report here the results of a questionnaire survey of consultant clinical geneticists in the United Kingdom to which we had an 81% response rate. In this questionnaire we asked about: (1) the nature of services currently offered to families with hearing impaired children, (2) what recurrence risks they quoted in isolated non-syndromic cases, and (3) what they might suggest for improving the range of genetic services available at present. We noted great variation both in these services and in the recurrence risks quoted in isolated cases. Based on the results of the questionnaire, we have proposed a protocol for the investigation of permanent childhood hearing impairment, which we believe to be both comprehensive and practical in an outpatient clinic setting. It is only by improving existing clinical and social understanding and knowledge of childhood hearing impairment that it will become possible to use recent molecular advances to develop comprehensive and consistent services for these families. (+info)
Human deafness dystonia syndrome is a mitochondrial disease. (2/1607)The human deafness dystonia syndrome results from the mutation of a protein (DDP) of unknown function. We show now that DDP is a mitochondrial protein and similar to five small proteins (Tim8p, Tim9p, Tim10p, Tim12p, and Tim13p) of the yeast mitochondrial intermembrane space. Tim9p, Tim10p, and Tim12p mediate the import of metabolite transporters from the cytoplasm into the mitochondrial inner membrane and interact structurally and functionally with Tim8p and Tim13p. DDP is most similar to Tim8p. Tim8p exists as a soluble 70-kDa complex with Tim13p and Tim9p, and deletion of Tim8p is synthetically lethal with a conditional mutation in Tim10p. The deafness dystonia syndrome thus is a novel type of mitochondrial disease that probably is caused by a defective mitochondrial protein-import system. (+info)
Contralateral deafness following unilateral suboccipital brain tumor surgery in a patient with large vestibular aqueduct--case report. (3/1607)A 68-year-old female developed contralateral deafness following extirpation of a left cerebellopontine angle epidermoid cyst. Computed tomography showed that large vestibular aqueduct was present. This unusual complication may have been caused by an abrupt pressure change after cerebrospinal fluid release, which was transmitted through the large vestibular aqueduct and resulted in cochlear damage. (+info)
Inner ear and kidney anomalies caused by IAP insertion in an intron of the Eya1 gene in a mouse model of BOR syndrome. (4/1607)A spontaneous mutation causing deafness and circling behavior was discovered in a C3H/HeJ colony of mice at the Jackson Laboratory. Pathological analysis of mutant mice revealed gross morphological abnormalities of the inner ear, and also dysmorphic or missing kidneys. The deafness and abnormal behavior were shown to be inherited as an autosomal recessive trait and mapped to mouse chromosome 1 near the position of the Eya1 gene. The human homolog of this gene, EYA1, has been shown to underly branchio-oto-renal (BOR) syndrome, an autosomal dominant disorder characterized by hearing loss with associated branchial and renal anomalies. Molecular analysis of the Eya1 gene in mutant mice revealed the insertion of an intracisternal A particle (IAP) element in intron 7. The presence of the IAP insertion was associated with reduced expression of the normal Eya1 message and formation of additional aberrant transcripts. The hypomorphic nature of the mutation may explain its recessive inheritance, if protein levels in homozygotes, but not heterozygotes, are below a critical threshold needed for normal developmental function. The new mouse mutation is designated Eya1(bor) to denote its similarity to human BOR syndrome, and will provide a valuable model for studying mutant gene expression and etiology. (+info)
Defective high-affinity thiamine transporter leads to cell death in thiamine-responsive megaloblastic anemia syndrome fibroblasts. (5/1607)We have investigated the cellular pathology of the syndrome called thiamine-responsive megaloblastic anemia (TRMA) with diabetes and deafness. Cultured diploid fibroblasts were grown in thiamine-free medium and dialyzed serum. Normal fibroblasts survived indefinitely without supplemental thiamine, whereas patient cells died in 5-14 days (mean 9.5 days), and heterozygous cells survived for more than 30 days. TRMA fibroblasts were rescued from death with 10-30 nM thiamine (in the range of normal plasma thiamine concentrations). Positive terminal deoxynucleotide transferase-mediated dUTP nick end-labeling (TUNEL) staining suggested that cell death was due to apoptosis. We assessed cellular uptake of [3H]thiamine at submicromolar concentrations. Normal fibroblasts exhibited saturable, high-affinity thiamine uptake (Km 400-550 nM; Vmax 11 pmol/min/10(6) cells) in addition to a low-affinity unsaturable component. Mutant cells lacked detectable high-affinity uptake. At 30 nM thiamine, the rate of uptake of thiamine by TRMA fibroblasts was 10-fold less than that of wild-type, and cells from obligate heterozygotes had an intermediate phenotype. Transfection of TRMA fibroblasts with the yeast thiamine transporter gene THI10 prevented cell death when cells were grown in the absence of supplemental thiamine. We therefore propose that the primary abnormality in TRMA is absence of a high-affinity thiamine transporter and that low intracellular thiamine concentrations in the mutant cells cause biochemical abnormalities that lead to apoptotic cell death. (+info)
Allele specific oligonucleotide analysis of the common deafness mutation 35delG in the connexin 26 (GJB2) gene. (6/1607)Despite the large number of genes that are expected to be involved in non-syndromal, recessive deafness, only a few have been cloned. One of these genes is GJB2, which encodes connexin 26. A frameshift mutation in this gene has been reported to be common in several populations and a carrier frequency of about 1 in 30 people has been detected in Italy and Spain. Mutation 35delG is difficult to detect because it lies within a stretch of six guanines flanked by thymines, so the deletion of one G does not create or destroy a restriction site and mutagenesis primers are not useful for this mutation. We have generated an allele specific oligonucleotide method that uses 12-mer oligonucleotides and easily discriminates between the normal and 35delG alleles. The method should permit a rapid analysis of this mutation in congenital cases (recessive or sporadic), including diagnosis and carrier detection of 35delG in the population. (+info)
A comparison of language achievement in children with cochlear implants and children using hearing aids. (7/1607)English language achievement of 29 prelingually deaf children with 3 or more years of cochlear implant (CI) experience was compared to the achievement levels of prelingually deaf children who did not have such CI experience. Language achievement was measured by the Rhode Island Test of Language Structure (RITLS), a measure of signed and spoken sentence comprehension, and the Index of Productive Syntax (IPSyn), a measure of expressive (signed and spoken) English grammar. When the CI users were compared with their deaf age mates who contributed to the norms of the RITLS, it was found that CI users achieved significantly better scores. Likewise, we found that CI users performed better than 29 deaf children who used hearing aids (HAs) with respect to English grammar achievement as indexed by the IPSyn. Additionally, we found that chronological age highly correlated with IPSyn levels only among the non-CI users, whereas length of CI experience was significantly correlated with IPSyn scores for CI users. Finally, clear differences between those with and without CI experience were found by 2 years of post-implant experience. These data provide evidence that children who receive CIs benefit in the form of improved English language comprehension and production. (+info)
Can routine information systems be used to monitor serious disability? (8/1607)OBJECTIVE: To determine whether reliable birth cohort prevalence rates of disabling conditions in early childhood can be obtained from child health information systems. DESIGN: Comparison of two sources of information on motor and sensory disabilities: from child health information systems held by health authorities, and a population register that uses multiple sources of ascertainment. SETTING: The counties of Oxfordshire, Buckinghamshire, and Northamptonshire. PARTICIPANTS: Children born to residents of the three counties between 1984 and 1989. RESULTS: Eight hundred and twenty children (6.0/1000 live births) were identified from the child health system as having one or more of the conditions, and 580 (4.2/1000 live births) were identified from the population register; however, only 284 children were identified by both sources. CONCLUSIONS: It is currently impossible to monitor trends in the prevalence rate of disabling disorders in childhood using the child health information systems. Agreement about ways of collecting, recording, and collating information on disability would be a useful step towards realising the full potential of these systems. (+info)
There are several types of deafness, including:
1. Conductive hearing loss: This type of deafness is caused by problems with the middle ear, including the eardrum or the bones of the middle ear. It can be treated with hearing aids or surgery.
2. Sensorineural hearing loss: This type of deafness is caused by damage to the inner ear or auditory nerve. It is typically permanent and cannot be treated with medication or surgery.
3. Mixed hearing loss: This type of deafness is a combination of conductive and sensorineural hearing loss.
4. Auditory processing disorder (APD): This is a condition in which the brain has difficulty processing sounds, even though the ears are functioning normally.
5. Tinnitus: This is a condition characterized by ringing or other sounds in the ears when there is no external source of sound. It can be a symptom of deafness or a separate condition.
There are several ways to diagnose deafness, including:
1. Hearing tests: These can be done in a doctor's office or at a hearing aid center. They involve listening to sounds through headphones and responding to them.
2. Imaging tests: These can include X-rays, CT scans, or MRI scans to look for any physical abnormalities in the ear or brain.
3. Auditory brainstem response (ABR) testing: This is a test that measures the electrical activity of the brain in response to sound. It can be used to diagnose hearing loss in infants and young children.
4. Otoacoustic emissions (OAE) testing: This is a test that measures the sounds produced by the inner ear in response to sound. It can be used to diagnose hearing loss in infants and young children.
There are several ways to treat deafness, including:
1. Hearing aids: These are devices that amplify sound and can be worn in or behind the ear. They can help improve hearing for people with mild to severe hearing loss.
2. Cochlear implants: These are devices that are implanted in the inner ear and can bypass damaged hair cells to directly stimulate the auditory nerve. They can help restore hearing for people with severe to profound hearing loss.
3. Speech therapy: This can help people with hearing loss improve their communication skills, such as speaking and listening.
4. Assistive technology: This can include devices such as captioned phones, alerting systems, and assistive listening devices that can help people with hearing loss communicate more effectively.
5. Medications: There are several medications available that can help treat deafness, such as antibiotics for bacterial infections or steroids to reduce inflammation.
6. Surgery: In some cases, surgery may be necessary to treat deafness, such as when there is a blockage in the ear or when a tumor is present.
7. Stem cell therapy: This is a relatively new area of research that involves using stem cells to repair damaged hair cells in the inner ear. It has shown promising results in some studies.
8. Gene therapy: This involves using genes to repair or replace damaged or missing genes that can cause deafness. It is still an experimental area of research, but it has shown promise in some studies.
9. Implantable devices: These are devices that are implanted in the inner ear and can help restore hearing by bypassing damaged hair cells. Examples include cochlear implants and auditory brainstem implants.
10. Binaural hearing: This involves using a combination of hearing aids and technology to improve hearing in both ears, which can help improve speech recognition and reduce the risk of falls.
It's important to note that the best treatment for deafness will depend on the underlying cause of the condition, as well as the individual's age, overall health, and personal preferences. It's important to work with a healthcare professional to determine the best course of treatment.
This type of hearing loss cannot be treated with medication or surgery, and it is usually permanent. However, there are various assistive devices and technology available to help individuals with sensorineural hearing loss communicate more effectively, such as hearing aids, cochlear implants, and FM systems.
There are several causes of sensorineural hearing loss, including:
1. Exposure to loud noises: Prolonged exposure to loud noises can damage the hair cells in the inner ear and cause permanent hearing loss.
2. Age: Sensorineural hearing loss is a common condition that affects many people as they age. It is estimated that one-third of people between the ages of 65 and 74 have some degree of hearing loss, and nearly half of those over the age of 75 have significant hearing loss.
3. Genetics: Some cases of sensorineural hearing loss are inherited and run in families.
4. Viral infections: Certain viral infections, such as meningitis or encephalitis, can damage the inner ear and cause permanent hearing loss.
5. Trauma to the head or ear: A head injury or a traumatic injury to the ear can cause sensorineural hearing loss.
6. Tumors: Certain types of tumors, such as acoustic neuroma, can cause sensorineural hearing loss by affecting the auditory nerve.
7. Ototoxicity: Certain medications, such as certain antibiotics, chemotherapy drugs, and aspirin at high doses, can be harmful to the inner ear and cause permanent hearing loss.
It is important to note that sensorineural hearing loss cannot be cured, but there are many resources available to help individuals with this condition communicate more effectively and improve their quality of life.
There are three main types of hearing loss: conductive, sensorineural, and mixed. Conductive hearing loss occurs when there is a problem with the middle ear and its ability to transmit sound waves to the inner ear. Sensorineural hearing loss occurs when there is damage to the inner ear or the auditory nerve, which can lead to permanent hearing loss. Mixed hearing loss is a combination of conductive and sensorineural hearing loss.
Symptoms of hearing loss may include difficulty hearing speech, especially in noisy environments, muffled or distorted sound, ringing or buzzing in the ears (tinnitus), and difficulty hearing high-pitched sounds. If you suspect you have hearing loss, it is important to seek medical advice as soon as possible, as early treatment can help improve communication and quality of life.
Hearing loss is diagnosed through a series of tests, including an audiometric test, which measures the softest sounds that can be heard at different frequencies. Treatment options for hearing loss include hearing aids, cochlear implants, and other assistive devices, as well as counseling and support to help manage the condition and improve communication skills.
Overall, hearing loss is a common condition that can have a significant impact on daily life. If you suspect you or someone you know may be experiencing hearing loss, it is important to seek medical advice as soon as possible to address any underlying issues and improve communication and quality of life.
Examples of syndromes include:
1. Down syndrome: A genetic disorder caused by an extra copy of chromosome 21 that affects intellectual and physical development.
2. Turner syndrome: A genetic disorder caused by a missing or partially deleted X chromosome that affects physical growth and development in females.
3. Marfan syndrome: A genetic disorder affecting the body's connective tissue, causing tall stature, long limbs, and cardiovascular problems.
4. Alzheimer's disease: A neurodegenerative disorder characterized by memory loss, confusion, and changes in personality and behavior.
5. Parkinson's disease: A neurological disorder characterized by tremors, rigidity, and difficulty with movement.
6. Klinefelter syndrome: A genetic disorder caused by an extra X chromosome in males, leading to infertility and other physical characteristics.
7. Williams syndrome: A rare genetic disorder caused by a deletion of genetic material on chromosome 7, characterized by cardiovascular problems, developmental delays, and a distinctive facial appearance.
8. Fragile X syndrome: The most common form of inherited intellectual disability, caused by an expansion of a specific gene on the X chromosome.
9. Prader-Willi syndrome: A genetic disorder caused by a defect in the hypothalamus, leading to problems with appetite regulation and obesity.
10. Sjogren's syndrome: An autoimmune disorder that affects the glands that produce tears and saliva, causing dry eyes and mouth.
Syndromes can be diagnosed through a combination of physical examination, medical history, laboratory tests, and imaging studies. Treatment for a syndrome depends on the underlying cause and the specific symptoms and signs presented by the patient.
This type of hearing loss can affect both children and adults and can range from mild to severe. The symptoms include difficulty hearing sounds on one side, difficulty localizing sounds, and difficulty understanding speech when there is background noise.
Unilateral hearing loss can be diagnosed with a variety of tests, including audiometry, otoscopy, and imaging studies. Treatment options vary depending on the cause of the hearing loss and may include hearing aids, cochlear implants, and surgery.
It is important to seek medical attention if you or your child experiences sudden or gradual hearing loss in one ear, as early diagnosis and treatment can help improve communication and quality of life.
The symptoms of bilateral hearing loss may include difficulty hearing speech, especially in noisy environments, difficulty understanding conversations when there is background noise, listening to loud music or watching television at a low volume, and experiencing ringing or buzzing sounds in the ears (tinnitus).
Bilateral hearing loss can be diagnosed with a thorough medical examination, including a physical examination of the ears, an audiometric test, and imaging tests such as CT or MRI scans.
Treatment options for bilateral hearing loss depend on the underlying cause and severity of the condition. Some possible treatment options include:
Hearing aids: These devices can amplify sounds and improve hearing ability.
Cochlear implants: These are electronic devices that are surgically implanted in the inner ear and can bypass damaged hair cells to directly stimulate the auditory nerve.
Assistive listening devices: These include devices such as FM systems, infrared systems, and alerting devices that can help individuals with hearing loss communicate more effectively.
Speech therapy: This can help improve communication skills and address any difficulties with language development.
Medications: Certain medications may be prescribed to treat underlying conditions that are contributing to the hearing loss, such as infections or excessive earwax.
Surgery: In some cases, surgery may be necessary to remove excessive earwax or to repair any damage to the middle ear bones.
The Usher syndromes are a group of rare genetic disorders that affect both hearing and vision. They are caused by mutations in specific genes and can be inherited in an autosomal recessive or X-linked manner. The syndromes are characterized by progressive retinal degeneration, hearing loss, and vestibular dysfunction.
Source: National Institute on Deafness and Other Communication Disorders (NIDCD)
Note: This is a medical definition, and the term "Usher Syndromes" is not commonly used in everyday conversation. It is used primarily in the medical field to describe this specific group of disorders.
The main symptoms of Wolfram syndrome include:
1. Diabetes insipidus (DI): A rare form of diabetes that affects the body's ability to regulate fluid levels.
2. Diabetes mellitus (DM): A common form of diabetes that affects blood sugar levels.
3. Optic atrophy: Degeneration of the nerve cells in the optic nerve, leading to vision loss and blindness.
4. Deafness: Hearing loss or complete deafness.
5. Hypogonadism: Low levels of sex hormones, which can lead to delayed or absent puberty.
6. Growth retardation: Delayed growth and development.
7. Intellectual disability: Cognitive impairment and learning difficulties.
8. Skeletal abnormalities: Abnormalities of the bones, such as short stature, scoliosis, or clubfoot.
9. Neurological symptoms: Such as seizures, ataxia, and peripheral neuropathy.
Wolfram syndrome is a rare and complex disorder, and there is currently no cure. Treatment focuses on managing the symptoms and preventing complications. Hormone replacement therapy may be used to treat hypogonadism, and insulin therapy may be used to manage diabetes. Physical therapy and occupational therapy can help improve mobility and independence. Regular monitoring by a multidisciplinary healthcare team is essential for managing the condition and improving the quality of life for individuals with Wolfram syndrome.
There are four types of Waardenburg Syndrome:
Type 1: This is the mildest form of the disorder and is characterized by subtle changes in skin and hair pigmentation and slight hearing loss. Individuals with this type typically have blue or grey eyes and a small amount of white hair.
Type 2: This type is more severe than Type 1 and is characterized by more pronounced pigmentation abnormalities, such as white patches on the skin and hair, as well as significant hearing loss. Individuals with this type often have intense blue or grey eyes and may experience developmental delays.
Type 3: This type is also severe and is characterized by a range of physical symptoms including hearing loss, pigmentation abnormalities, and skeletal deformities such as short stature or joint contractures. Individuals with this type often have unique facial features, such as a broad forehead, narrow eyes, and a long nose.
Type 4: This is the most severe form of Waardenburg syndrome and is characterized by profound hearing loss, significant pigmentation abnormalities, and multiple congenital anomalies such as heart defects or digestive system problems. Individuals with this type often have a short life expectancy and may require extensive medical care throughout their lives.
Inheritance Pattern: Waardenburg syndrome is inherited in an autosomal dominant pattern, meaning that a single copy of the mutated gene is enough to cause the condition. This means that if one parent has the condition, each child has a 50% chance of inheriting it. However, some forms of the condition may be more severe than others and may require specialized medical care.
Treatment and Management: There is no cure for Waardenburg syndrome, but various treatments can help manage its symptoms. Hearing aids or cochlear implants can help improve hearing, while surgery or physical therapy can help correct skeletal deformities. Regular monitoring by a medical professional is also important to ensure that any related health issues are addressed promptly.
In conclusion, Waardenburg syndrome is a rare genetic disorder that affects the development of pigmentation and hearing in individuals. It can range from mild to severe forms, each with distinct physical characteristics and medical needs. With proper management and care, individuals with Waardenburg syndrome can lead fulfilling lives despite the challenges posed by this condition.
The primary symptom of hypoparathyroidism is low blood calcium levels, which can lead to tingling or numbness in the fingers and toes, muscle cramps, twitching, and spasms. Other signs may include brittle nails, thinning hair, and poor wound healing. In severe cases, hypoparathyroidism can cause seizures, coma, and even death.
Hypoparathyroidism is usually diagnosed through a combination of physical examination, blood tests, and imaging studies such as ultrasound or CT scans. Treatment typically involves replacing calcium and vitamin D hormones, which can help manage symptoms and prevent complications. In some cases, medications that stimulate the parathyroid glands may be prescribed to increase calcium production. Surgery may be necessary in cases where the condition is caused by a tumor or other structural abnormality.
Prognosis for hypoparathyroidism varies depending on the underlying cause and severity of the condition. With appropriate treatment, many people with hypoparathyroidism can lead normal lives, but some may experience persistent symptoms or complications such as osteoporosis, kidney stones, or cognitive impairment.
Here are some of the main points about Jervell-Lange Nielsen Syndrome:
1. Rare genetic disorder: Jervell-Lange Nielsen Syndrome is a rare genetic disorder that affects the development of the nervous system.
2. Progressive muscle weakness: The syndrome is characterized by progressive muscle weakness, which can make it difficult for individuals to walk and perform other daily activities.
3. High risk of sudden death: Individuals with Jervell-Lange Nielsen Syndrome have a high risk of sudden death due to cardiac arrhythmias, which can be triggered by stress, exercise, or other factors.
4. Caused by genetic mutations: The syndrome is caused by mutations in genes that code for proteins involved in the development and maintenance of nerve cells.
5. No cure: There is no cure for Jervell-Lange Nielsen Syndrome, but treatment options are available to manage symptoms and prevent complications.
6. Poor prognosis: The prognosis for individuals with this syndrome is generally poor, with most individuals dying before the age of 20 due to cardiac or respiratory failure.
7. Other names: Jervell-Lange Nielsen Syndrome is also known as type 1 long QT syndrome, which refers to the abnormal heart rhythm that is a hallmark of the condition.
8. Rare: The syndrome is rare, with an estimated prevalence of 1 in 200,000 to 1 in 500,000 individuals worldwide.
9. Inherited in an autosomal recessive pattern: Jervell-Lange Nielsen Syndrome is inherited in an autosomal recessive pattern, meaning that a child must inherit two copies of the mutated gene (one from each parent) to develop the condition.
10. Diagnostic criteria: The diagnosis of Jervell-Lange Nielsen Syndrome is based on a combination of clinical features, including progressive muscle weakness, high risk of sudden death, and characteristic electrocardiogram (ECG) findings. Genetic testing can also be used to confirm the diagnosis.
I hope this list helps you understand Jervell-Lange Nielsen Syndrome better! Let me know if you have any other questions.
Some common types of cochlear diseases include:
1. Cochlear implants: These are electronic devices that are surgically implanted in the inner ear to bypass damaged hair cells and directly stimulate the auditory nerve. Cochlear implants can help restore hearing in individuals with severe to profound sensorineural hearing loss.
2. Meniere's disease: This is a disorder of the inner ear that can cause vertigo, tinnitus, hearing loss, and a feeling of fullness in the affected ear. The exact cause of Meniere's disease is not known, but it is thought to be related to an abnormal accumulation of fluid in the inner ear.
3. Ototoxicity: This refers to damage to the inner ear or auditory nerve caused by exposure to certain medications, chemicals, or other substances. Ototoxicity can result in hearing loss, tinnitus, and balance problems.
4. Presbycusis: This is age-related hearing loss that affects the inner ear and is a common condition in older adults. Presbycusis can cause gradual hearing loss over time and may be treated with hearing aids or other assistive devices.
5. Sudden sensorineural hearing loss (SSHL): This is a sudden and severe loss of hearing that occurs within a few days or weeks and can be caused by a variety of factors, including viral infections, head trauma, and certain medications. SSHL is a medical emergency and requires prompt treatment to improve the chances of recovering some or all of the lost hearing.
Overall, cochlear diseases can have a significant impact on an individual's quality of life, affecting their ability to communicate and interact with others. Fortunately, many of these conditions can be treated with hearing aids, medications, or surgery, and there are also a variety of assistive devices and strategies that can help individuals manage their symptoms and improve their communication skills.
Some common examples of vestibular diseases include:
1. Benign paroxysmal positional vertigo (BPPV): A condition that causes brief episodes of vertigo triggered by changes in head position.
2. Labyrinthitis: An inner ear infection that causes vertigo, hearing loss, and tinnitus (ringing in the ears).
3. Vestibular migraine: A type of migraine that causes vertigo, along with headaches and other symptoms.
4. Meniere's disease: A disorder of the inner ear that causes vertigo, tinnitus, hearing loss, and a feeling of fullness in the affected ear.
5. Acoustic neuroma: A benign tumor that grows on the nerve that connects the inner ear to the brain, causing symptoms such as vertigo, hearing loss, and tinnitus.
6. Superior canal dehiscence syndrome: A condition in which the bony covering of the superior canal in the inner ear is thin or absent, leading to symptoms such as vertigo, hearing loss, and sound sensitivity.
7. Perilymph fistula: A tear or defect in the membrane that separates the middle ear from the inner ear, causing symptoms such as vertigo, hearing loss, and tinnitus.
8. Ototoxicity: Damage to the inner ear caused by exposure to certain medications or chemicals, leading to symptoms such as vertigo, hearing loss, and tinnitus.
Diagnosis of vestibular diseases typically involves a combination of medical history, physical examination, and specialized tests such as the Electronystagmography (ENG) or Vestibular Function Tests (VFT). Treatment options vary depending on the underlying cause of the symptoms, but may include medications, vestibular rehabilitation therapy, or surgery.
Types of Hearing Disorders:
1. Conductive hearing loss: This type of hearing loss is caused by a problem with the middle ear, including the eardrum or the bones of the middle ear. It can be treated with hearing aids or surgery.
2. Sensorineural hearing loss: This type of hearing loss is caused by damage to the inner ear or the auditory nerve. It is permanent and cannot be treated with medicine or surgery.
3. Mixed hearing loss: This type of hearing loss is a combination of conductive and sensorineural hearing loss.
4. Tinnitus: This is the perception of ringing, buzzing, or other sounds in the ears when there is no external source of the sound. It can be caused by exposure to loud noises, age, or certain medications.
5. Balance disorders: These are conditions that affect the balance center in the inner ear or the brain, causing dizziness, vertigo, and other symptoms.
Causes of Hearing Disorders:
1. Genetics: Some hearing disorders can be inherited from parents or grandparents.
2. Age: As we age, our hearing can decline due to wear and tear on the inner ear.
3. Exposure to loud noises: Prolonged exposure to loud sounds, such as music or machinery, can damage the hair cells in the inner ear and lead to hearing loss.
4. Infections: Certain infections, such as otitis media (middle ear infection), can cause hearing loss if left untreated.
5. Certain medications: Some medications, such as certain antibiotics, chemotherapy drugs, and aspirin at high doses, can be harmful to the inner ear and cause hearing loss.
Symptoms of Hearing Disorders:
1. Difficulty hearing or understanding speech, especially in noisy environments.
2. Ringing, buzzing, or other sounds in the ears (tinnitus).
3. Vertigo or dizziness.
4. Feeling of fullness or pressure in the ears.
5. Hearing loss that worsens over time.
Diagnosis and Treatment of Hearing Disorders:
1. Medical history and physical examination.
2. Audiometry test to measure hearing threshold and speech discrimination.
3. Otoscopy to examine the outer ear and ear canal.
4. Tympanometry to assess the middle ear function.
5. Otoacoustic emissions testing to evaluate the inner ear function.
Treatment options for hearing disorders depend on the underlying cause and may include:
1. Hearing aids or cochlear implants to improve hearing.
2. Medications to treat infections or reduce tinnitus.
3. Surgery to remove earwax, repair the eardrum, or address middle ear problems.
4. Balance rehabilitation exercises to manage vertigo and dizziness.
5. Cognitive therapy to improve communication skills and address psychological effects of hearing loss.
Prevention and Management of Hearing Disorders:
1. Avoiding loud noises and taking regular breaks in noisy environments.
2. Wearing earplugs or earmuffs when exposed to loud sounds.
3. Getting regular hearing checkups and addressing any hearing issues promptly.
4. Managing chronic conditions, such as diabetes and hypertension, that can contribute to hearing loss.
5. Encouraging open communication with family members and healthcare providers about hearing difficulties.
Symptoms of conductive hearing loss may include:
* Difficulty hearing soft sounds
* Muffled or distorted sound
* Ringing or other noises in the affected ear
* Difficulty understanding speech, especially in noisy environments
Causes of conductive hearing loss can include:
* Middle ear infections (otitis media)
* Eardrum perforation or tearing
* Tubal erosion or narrowing
* Ossicular anomalies or abnormalities
* Certain head or neck injuries
* Tumors or cysts in the middle ear
Diagnosis of conductive hearing loss typically involves a physical examination and a series of tests, including:
* Otoscopy (examination of the outer ear and eardrum)
* Tympanometry (measurement of the movement of the eardrum)
* Acoustic reflex threshold testing (assessment of the acoustic reflex, which is a normal response to loud sounds)
* Otoacoustic emissions testing (measurement of the sounds produced by the inner ear in response to sound waves)
Treatment for conductive hearing loss depends on the underlying cause and may include:
* Antibiotics for middle ear infections
* Tubes inserted into the eardrum to drain fluid and improve air flow
* Surgery to repair or replace damaged ossicles or other middle ear structures
* Hearing aids or cochlear implants to amplify sound waves and improve hearing.
Some common examples of labyrinth diseases include:
1. Meniere's disease: This is a disorder of the inner ear that causes episodes of vertigo (spinning), tinnitus, hearing loss, and a feeling of fullness in the affected ear.
2. Acoustic neuroma: This is a benign tumor that grows on the nerve that connects the inner ear to the brain. It can cause hearing loss, tinnitus, balance difficulties, and facial weakness or paralysis.
3. Ototoxicity: This is damage to the inner ear caused by certain medications or chemicals. It can lead to hearing loss, tinnitus, and balance difficulties.
4. Presbycusis: This is age-related hearing loss that affects the inner ear and can cause gradual hearing loss over time.
5. Benign paroxysmal positional vertigo (BPPV): This is a condition that causes brief episodes of vertigo triggered by changes in head position. It is caused by small crystals in the inner ear becoming dislodged and moving into the canals of the inner ear.
6. Labyrinthitis: This is an inner ear infection that causes hearing loss, tinnitus, and balance difficulties.
7. Vestibular migraine: This is a type of migraine headache that causes vertigo, hearing loss, and tinnitus.
These are just a few examples of labyrinth diseases. There are many other conditions that can affect the inner ear and cause similar symptoms. If you suspect you may have a labyrinth disease, it is important to seek medical attention to receive an accurate diagnosis and appropriate treatment.
Symptoms of ichthyosis can include:
* Thickened, scaly skin on the arms, legs, back, and chest
* Redness and itching
* Cracking and splitting of the skin
* Increased risk of infection
* Respiratory problems
Treatment for ichthyosis typically involves the use of topical creams and ointments to help soften and hydrate the skin, as well as oral medications to reduce inflammation and itching. In severe cases, phototherapy or systemic corticosteroids may be necessary.
In addition to these medical treatments, there are also several home remedies and lifestyle modifications that can help manage the symptoms of ichthyosis. These include:
* Moisturizing regularly with a fragrance-free moisturizer
* Avoiding harsh soaps and cleansers
* Using lukewarm water when showering or bathing
* Applying cool compresses to the skin to reduce redness and inflammation
* Wearing loose, breathable clothing to avoid irritating the skin
* Protecting the skin from extreme temperatures and environmental stressors.
Some examples of multiple abnormalities include:
1. Multiple chronic conditions: An individual may have multiple chronic conditions such as diabetes, hypertension, arthritis, and heart disease, which can affect their quality of life and increase their risk of complications.
2. Congenital anomalies: Some individuals may be born with multiple physical abnormalities or birth defects, such as heart defects, limb abnormalities, or facial deformities.
3. Mental health disorders: Individuals may experience multiple mental health disorders, such as depression, anxiety, and bipolar disorder, which can impact their cognitive functioning and daily life.
4. Neurological conditions: Some individuals may have multiple neurological conditions, such as epilepsy, Parkinson's disease, and stroke, which can affect their cognitive and physical functioning.
5. Genetic disorders: Individuals with genetic disorders, such as Down syndrome or Turner syndrome, may experience a range of physical and developmental abnormalities.
The term "multiple abnormalities" is often used in medical research and clinical practice to describe individuals who have complex health needs and require comprehensive care. It is important for healthcare providers to recognize and address the multiple needs of these individuals to improve their overall health outcomes.
There are several types of ataxia, each with different symptoms and causes. Some common forms of ataxia include:
1. Spinocerebellar ataxia (SCA): This is the most common form of ataxia and is caused by a degeneration of the cerebellum and spinal cord. It can cause progressive weakness, loss of coordination, and difficulty with speaking and swallowing.
2. Friedreich's ataxia: This is the second most common form of ataxia and is caused by a deficiency of vitamin E in the body. It can cause weakness in the legs, difficulty walking, and problems with speech and language.
3. Ataxia-telangiectasia (AT): This is a rare form of ataxia that is caused by a gene mutation. It can cause progressive weakness, loss of coordination, and an increased risk of developing cancer.
4. Acute cerebellar ataxia: This is a sudden and temporary form of ataxia that can be caused by a variety of factors such as infections, injuries, or certain medications.
5. Drug-induced ataxia: Certain medications can cause ataxia as a side effect.
6. Vitamin deficiency ataxia: Deficiencies in vitamins such as vitamin B12 or folate can cause ataxia.
7. Metabolic disorders: Certain metabolic disorders such as hypothyroidism, hyperthyroidism, and hypoglycemia can cause ataxia.
8. Stroke or brain injury: Ataxia can be a result of a stroke or brain injury.
9. Multiple system atrophy (MSA): This is a rare progressive neurodegenerative disorder that can cause ataxia, parkinsonism, and autonomic dysfunction.
10. Spinocerebellar ataxia (SCA): This is a group of rare genetic disorders that can cause progressive cerebellar ataxia, muscle wasting, and other signs and symptoms.
It's important to note that this is not an exhaustive list and there may be other causes of ataxia not mentioned here. If you suspect you or someone you know may have ataxia, it is important to consult a healthcare professional for proper diagnosis and treatment.
1. Hypothyroidism: An underactive thyroid gland can cause the gland to become enlarged as it tries to produce more hormones to compensate for the lack of production.
2. Hyperthyroidism: An overactive thyroid gland can also cause the gland to become enlarged as it produces excessive amounts of hormones.
3. Thyroid nodules: These are abnormal growths within the thyroid gland that can cause the gland to become enlarged.
4. Thyroiditis: This is an inflammation of the thyroid gland that can cause it to become enlarged.
5. Iodine deficiency: Iodine is essential for the production of thyroid hormones, and a lack of iodine in the diet can cause the gland to become enlarged as it tries to produce more hormones.
6. Pituitary gland problems: The pituitary gland, located at the base of the brain, regulates the production of thyroid hormones. Problems with the pituitary gland can cause the thyroid gland to become enlarged.
7. Genetic conditions: Some genetic conditions, such as familial goiter, can cause the thyroid gland to become enlarged.
Symptoms of goiter may include:
* A noticeable lump in the neck
* Difficulty swallowing or breathing
* Hoarseness or vocal cord paralysis
* Weight gain
* Cold intolerance
Goiter can be diagnosed through a physical examination, blood tests to measure thyroid hormone levels, and imaging studies such as ultrasound or radionuclide scans to evaluate the size and function of the gland. Treatment options for goiter depend on the underlying cause and may include medication, surgery, or radioactive iodine therapy.
Symptoms of hereditary nephritis may include blood in the urine, proteinuria (excess protein in the urine), edema (swelling), high blood pressure, and kidney failure. The disorder can be diagnosed through blood tests, such as a viral load or genetic testing, and imaging studies, such as ultrasound or CT scans.
There is no cure for hereditary nephritis, but treatment options are available to manage the symptoms and slow the progression of the disease. Treatment may include medications to control blood pressure, reduce proteinuria, and prevent further kidney damage. In severe cases, dialysis or a kidney transplant may be necessary.
Bulbar palsy, progressive refers to a condition where there is a gradual loss of muscle function in the face, tongue, and throat due to damage to the brainstem. This condition is also known as progressive bulbar palsy (PBP).
The brainstem is responsible for controlling many of the body's automatic functions, including breathing, heart rate, and swallowing. When the brainstem is damaged, it can lead to a range of symptoms, including weakness or paralysis of the muscles in the face, tongue, and throat.
The symptoms of progressive bulbar palsy may include:
* Difficulty speaking or slurred speech
* Weakness or paralysis of the facial muscles
* Difficulty swallowing (dysphagia)
* Weight loss due to difficulty eating and drinking
* Fatigue and weakness
* Decreased reflexes
Progressive bulbar palsy can be caused by a variety of conditions, including:
* Brainstem stroke or bleeding
* Brain tumors
* Multiple sclerosis
* Amyotrophic lateral sclerosis (ALS)
* Other neurodegenerative disorders
There is no cure for progressive bulbar palsy, but treatment may include:
* Speech therapy to improve communication skills
* Swallowing therapy to reduce the risk of choking or pneumonia
* Physical therapy to maintain muscle strength and function
* Medications to manage symptoms such as pain, weakness, or fatigue
The prognosis for progressive bulbar palsy is generally poor, with many individuals experiencing significant decline in their quality of life and eventually succumbing to the disease. However, the rate of progression can vary greatly depending on the underlying cause of the condition.
1. Retinal coloboma: A condition where a hole or gap in the retina, the light-sensitive tissue at the back of the eye, can cause vision loss or blindness.
2. Cerebral coloboma: A condition where a part of the brain is missing or underdeveloped, which can result in intellectual disability, seizures, and other neurological symptoms.
3. Coloboma of the eye: A condition where the iris or optic nerve is not properly formed, leading to vision problems such as amblyopia (lazy eye) or strabismus (crossed eyes).
Note: Coloboma is a relatively rare condition and can be diagnosed through imaging tests such as ultrasound, CT scan, or MRI. Treatment options vary depending on the location and severity of the defect, and may include surgery, medication, or other interventions to manage associated symptoms.
The term "keratoderma" comes from the Greek words "keras," meaning "horn," and "derma," meaning "skin." It refers to the characteristic thickening of the skin that is seen in these disorders. The term "palmoplantar" refers to the fact that the condition affects the palms and soles of the feet.
Keratoderma, palmoplantar can be caused by a variety of genetic mutations, including autosomal dominant, autosomal recessive, and X-linked inheritance patterns. The disorder is usually diagnosed based on the appearance of the skin and may require a biopsy or genetic testing to confirm the diagnosis.
Treatment for keratoderma, palmoplantar typically focuses on managing the symptoms and preventing complications. This may include topical medications, physical therapy, and lifestyle modifications such as wearing gloves or protective footwear to reduce friction and irritation. In some cases, surgery may be necessary to remove thickened skin or repair damaged tissue.
Overall, keratoderma, palmoplantar is a rare and debilitating condition that can significantly impact an individual's quality of life. With proper diagnosis and management, however, it is possible to alleviate symptoms and improve functioning.
Deafness in Tunisia
Deafness in Ghana
Deafness in Haiti
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Deafness in France
Microcephaly deafness syndrome
Deafness Research UK
Deafness in Thailand
Deafness in India
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Models of deafness
Deafness in Nicaragua
Deafness in Poland
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Diabetes and deafness
Deafness in Ireland
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Perception of Deaf and Dumb - Deafness
North Jersey umpire doesn't let deafness deter him
Sensorineural deafness: MedlinePlus Medical Encyclopedia
Browsing by Subject "Deafness"
Ethics in Mental Health and Deafness
Hearing Loss Stories | Single-Sided Deafness
WHO EMRO | Deafness | Health topics
It's Not The Same Old Deafness | Jane Madell |hearinghealthmatters.org/hearingandkids
Nonsyndromic Deafness (LOXHD1-related) - Norton & Elaine Sarnoff Center for Jewish Genetics
Listen Technologies ListenLoop Systems Installed at The National Institute on Deafness and Other Communication Disorders (NIDCD...
New Zealand Deafness Notification Database
Deafness in Australian Cattle Dogs associated to QTL on chromosome 20 in genome-wide association study analyses. | Profiles RNS
President's Council on Deafness - The Deaf President Now (DPN) Protest | Gallaudet University
Deafness Variation Database
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jebo copyrights: DEAFNESS BY NOISE
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capital-hill-deafness-resource-person - DeafDigest
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- Foster, a research associate in Mustafa Tekin's lab at the University of Miami's Hussman Institute for Human Genomics, is involved in the hunt for the remaining genes responsible for congenital forms of deafness.This area of research is a good fit for Foster. (nih.gov)
- Congenital nerve deafness is due to arrested development of the otocyst during the second and third fetal months or to prenatal degeneration of the organ of Corti. (nih.gov)
- Congenital nerve deafness cannot be corrected medically or surgically. (nih.gov)
Experience with deafness2
- In its final recommendation, the PCD named I. King Jordan and Harvey Corson as the only acceptable candidates, noting that none of the hearing candidates had any experience with deafness or deaf people. (gallaudet.edu)
- Even lesser well known is Locke's own experience with deafness. (rubenverwaal.com)
- NIH-funded global research has uncovered more than 50 recessive genes tied to hereditary deafness, bringing science closer to better treatments. (nih.gov)
- The inventors have discovered that dominant and recessive mutations in TMC1 underlie two forms of hereditary deafness, known as DFNA36 and DFNB7/11. (nih.gov)
- Sensorineural deafness is a type of hearing loss. (medlineplus.gov)
- That has provided research opportunities for NIDCD-sponsored scientists and their collaborators to identify deafness-causing genetic mutations in Pakistan, the West Bank, Israel, India, Indonesia, Costa Rica and Brazil. (nih.gov)
- Only 10% of deaf people have deaf children, although 60% of deaf people do not know the cause of their deafness, and much of that may be attributed to genetic issues which could increase the statistic above. (gatecommunications.org)
- There is of course genetic deafness. (gatecommunications.org)
- Ethics and Mental Health in Deafness also features a chapter on genetic counseling and testing for deafness by Kathleen Arnos. (gallaudet.edu)
- The Molecular Otolaryngology & Renal Research Lab (MORL) Deafness Variation Database (DVD) provides a comprehensive guide to genetic variation in genes known to be associated with deafness. (deafnessvariationdatabase.org)
- The DVD v9 includes all known genetic variants present in 223 deafness-associated genes that are included on OtoSCOPE® , the MORL's comprehensive genetic deafness screening platform . (deafnessvariationdatabase.org)
- Genetic and metabolic deafness / Bruce W. Konigsmark, Robert J. Gorlin. (who.int)
- Genes are behind much of the deafness that exists worldwide and research supported by the National Institute on Deafness and Other Communication Disorders (NIDCD) - in large part from global health studies - has uncovered about half of the 50 known genes in which mutations cause deafness inherited as a recessive trait. (nih.gov)
- All of the genes that we've identified in Pakistan are the same genes that cause deafness worldwide, every single one of them," Friedman said. (nih.gov)
- He has subsequently collaborated on global projects that have pinpointed genes culpable in deafness and other disorders as well. (nih.gov)
- Collaborating in Costa Rica then Israel and Palestine, her teams identified several genes for deafness in large families. (nih.gov)
- Deaf people who are genetically deaf can have either a pair of recessive genes causing their deafness or a dominant gene passed down from their parents. (gatecommunications.org)
- Many Hearing parents do not realize that they are carrying genes for deafness until they have Deaf children. (gatecommunications.org)
- Please take some time to review the sources we use for annotation as well as our landmark paper " Genomic Landscape and Mutational Signatures of Deafness-Associated Genes " for more information regarding this work. (deafnessvariationdatabase.org)
- Dominant and recessive deafness caused by mutations of a novel gene, TMC1, required for cochlear hair-cell function. (nih.gov)
- 1 Laboratory of Molecular Genetics, National Institute on Deafness and Other Communication Disorders, Porter Neuroscience Research Center, National Institutes of Health, Bethesda, MD 20892, USA. (nih.gov)
- Nerve deafness is not uncommon. (gatecommunications.org)
- Dr. Debara L. Tucci is the Director of the National Institute on Deafness and Other Communication Disorders. (medlineplus.gov)
- As Director of the National Institute on Deafness and Other Communication Disorders (NIDCD), it's her job to bring together scientists and other experts who are trying to improve our understanding of these conditions. (medlineplus.gov)
- This data-driven discovery could provide novel insights into prevention, diagnosis, and treatment for individuals with deafness and communication disorders. (medlineplus.gov)
- National Institute on Deafness and Other Communication Disorders website. (medlineplus.gov)
- Cooperation and collaboration with professional, academic, commercial, voluntary, and philanthropic organizations concerned with research and training that is related to deafness and other communication disorders, disease prevention and health promotion, and the biomedical and behavioral issues associated with communication impairments or disorders. (nih.gov)
- February 15, 1989 - The secretary of the Department of Health and Human Services (HHS) establishes the National Deafness and Other Communication Disorders Advisory Council. (nih.gov)
- April 4, 1991 - The Deafness and Other Communication Disorders Interagency Coordinating Committee (ICC) is established to foster collaboration among federal agencies and facilitate the sharing of information on their efforts in the field of deafness and other communication disorders. (nih.gov)
- SMALL GRANT PROGRAM FOR THE NATIONAL INSTITUTE ON DEAFNESS AND OTHERCOMMUNICATION DISORDERS NIH GUIDE, Volume 22, Number 39, October 29, 1993 PA NUMBER: PAR-94-009 P.T. 34 Keywords: Communicative Disorders, Hearing Communicative Disorders, Speech Language Acquisition & Development 0775017 National Institute on Deafness and Other Communication Disorders PURPOSE This program announcement supersedes all previously issued announcements for the National Institute on Deafness and Other Communication Disorders (NIDCD) Small Grant Program. (nih.gov)
- BLUFFDALE, UT, May 12, 2015 - The National Institute on Deafness and Other Communication Disorders (NIDCD) , in Bethesda, Maryland, is one of the world's leading institutions for research into hearing and auditory health issues. (listentech.com)
- 10 Auditory Development and Restoration Program, National Institute on Deafness and Other Communication Disorders, National Institutes of Health, Bethesda, MD 20892, USA. (nih.gov)
- On September 3, 2019, Debra Tucci (right) joined the NIH family as director of the National Institute on Deafness and Other Communication Disorders (NIDCD). (nih.gov)
- Over the coming months, and with the guidance of NIDCD staff and our advisory committees-the National Deafness and Other Communication Disorders Advisory Council and the Board of Scientific Counselors-we will examine our current portfolio and priorities, opportunities for growth and partnership, and the needs of our targeted populations and constituencies to craft a strategic plan for the future. (nih.gov)
- I look forward to working with all within the NIDCD-in partnership with those affected by deafness and other communication disorders and with our scientific community-to achieve these goals. (nih.gov)
- Debara L. Tucci, M.D., M.S., M.B.A. , became director of the National Institute on Deafness and Other Communication Disorders (NIDCD), part of the National Institutes of Health, on September 3, 2019. (nih.gov)
- The New Zealand Deafness Notification Database (DND) contains information on children and young people under the age of 19 years who have been diagnosed with a permanent hearing loss in one or both ears. (audiology.org.nz)
- Children with hearing loss and deafness in developing countries rarely receive any schooling. (who.int)
- King, a long-term Fogarty and NIDCD grantee, has investigated deafness globally since 1990. (nih.gov)
- This was the case in Perrault syndrome, where a deafness gene found in three Pakistani families was subsequently linked to ovarian failure, abnormal organ development, movement disorders, learning disabilities and peripheral neuropathy. (nih.gov)
- This test is based on an association study in which multiple DNA variants in close proximity to the usp31 gene on chromosome 6 were discovered that are related to Early Adult Onset Deafness (EAOD) in Border Collies. (combibreed.com)
- It is described in the literature that the presence of deafness in the pedigree greatly increases the chances that these risk variants are associated with the causal mutation for EAOD, which has not been discovered yet. (combibreed.com)
- This paper proposes that the renewed focus on the fluids brought about a new understanding of auditory perception, which reconstructed hearing and deafness not in terms of a dichotomy, but in terms of a grading scale. (rubenverwaal.com)
- Fascinated as I was in watching them, and having no understanding of deafness, the perception I formed was that these children were mentally deficient, simply because they couldn't talk like me and my friends. (bellaonline.com)
- Helping deaf kids hear better does not mean we are rejecting deafness or rejecting our children. (hearinghealthmatters.org)
- Many people, me included, who went deaf later in life, find that deafness characteristics creep into their speech simply because they cannot clearly hear what they are saying. (bellaonline.com)
- This talk discusses hearing disability in early modern science and presents Enlightenment medicine as part of a profound shift in thinking about deafness. (rubenverwaal.com)
- In the early 1990s, Friedman's research took him to Bali, Indonesia, to a village where deafness was so common that hearing people and non-hearing alike used a unique sign language. (nih.gov)
- Gate wants to introduce you to the beauty of the Deaf Culture , the diversity of people with deafness, and provide you with resources that can help you whether you are interacting with the Deaf for the first time, have a Deaf child, involved in a Hearing/Deaf relationship, or just wanting resources to further your understanding of the Deaf World. (gatecommunications.org)
- A lot of people who still argue that all deaf kids should learn to sign are not recognizing that "it is not the same old deafness. (hearinghealthmatters.org)
- Deafness is a mystery to individuals not involved in the Deaf Community. (gatecommunications.org)
- Early Adult Onset Deafness (EAOD) is a hearing disease in Border Collies and has an early onset at 3-5 years. (combibreed.com)
- Subscribe for free weekly updates from this Deafness site. (bellaonline.com)
- In this way, deafness induces an atypical lateralization for memory localization, while the acquisition of the sign language improves the performance in visuospatial tasks. (bvsalud.org)