Twelve pairs of nerves that carry general afferent, visceral afferent, special afferent, somatic efferent, and autonomic efferent fibers.
Disorders of one or more of the twelve cranial nerves. With the exception of the optic and olfactory nerves, this includes disorders of the brain stem nuclei from which the cranial nerves originate or terminate.
Dysfunction of one or more cranial nerves causally related to a traumatic injury. Penetrating and nonpenetrating CRANIOCEREBRAL TRAUMA; NECK INJURIES; and trauma to the facial region are conditions associated with cranial nerve injuries.
Benign and malignant neoplasms that arise from one or more of the twelve cranial nerves.
Diseases of the sixth cranial (abducens) nerve or its nucleus in the pons. The nerve may be injured along its course in the pons, intracranially as it travels along the base of the brain, in the cavernous sinus, or at the level of superior orbital fissure or orbit. Dysfunction of the nerve causes lateral rectus muscle weakness, resulting in horizontal diplopia that is maximal when the affected eye is abducted and ESOTROPIA. Common conditions associated with nerve injury include INTRACRANIAL HYPERTENSION; CRANIOCEREBRAL TRAUMA; ISCHEMIA; and INFRATENTORIAL NEOPLASMS.
The 7th cranial nerve. The facial nerve has two parts, the larger motor root which may be called the facial nerve proper, and the smaller intermediate or sensory root. Together they provide efferent innervation to the muscles of facial expression and to the lacrimal and SALIVARY GLANDS, and convey afferent information for TASTE from the anterior two-thirds of the TONGUE and for TOUCH from the EXTERNAL EAR.
Diseases of the oculomotor nerve or nucleus that result in weakness or paralysis of the superior rectus, inferior rectus, medial rectus, inferior oblique, or levator palpebrae muscles, or impaired parasympathetic innervation to the pupil. With a complete oculomotor palsy, the eyelid will be paralyzed, the eye will be in an abducted and inferior position, and the pupil will be markedly dilated. Commonly associated conditions include neoplasms, CRANIOCEREBRAL TRAUMA, ischemia (especially in association with DIABETES MELLITUS), and aneurysmal compression. (From Adams et al., Principles of Neurology, 6th ed, p270)
A nerve which originates in the lumbar and sacral spinal cord (L4 to S3) and supplies motor and sensory innervation to the lower extremity. The sciatic nerve, which is the main continuation of the sacral plexus, is the largest nerve in the body. It has two major branches, the TIBIAL NERVE and the PERONEAL NERVE.
The 9th cranial nerve. The glossopharyngeal nerve is a mixed motor and sensory nerve; it conveys somatic and autonomic efferents as well as general, special, and visceral afferents. Among the connections are motor fibers to the stylopharyngeus muscle, parasympathetic fibers to the parotid glands, general and taste afferents from the posterior third of the tongue, the nasopharynx, and the palate, and afferents from baroreceptors and CHEMORECEPTOR CELLS of the carotid sinus.
The 3d cranial nerve. The oculomotor nerve sends motor fibers to the levator muscles of the eyelid and to the superior rectus, inferior rectus, and inferior oblique muscles of the eye. It also sends parasympathetic efferents (via the ciliary ganglion) to the muscles controlling pupillary constriction and accommodation. The motor fibers originate in the oculomotor nuclei of the midbrain.
The nerves outside of the brain and spinal cord, including the autonomic, cranial, and spinal nerves. Peripheral nerves contain non-neuronal cells and connective tissue as well as axons. The connective tissue layers include, from the outside to the inside, the epineurium, the perineurium, and the endoneurium.
Traumatic injuries to the HYPOGLOSSAL NERVE.
The 5th and largest cranial nerve. The trigeminal nerve is a mixed motor and sensory nerve. The larger sensory part forms the ophthalmic, mandibular, and maxillary nerves which carry afferents sensitive to external or internal stimuli from the skin, muscles, and joints of the face and mouth and from the teeth. Most of these fibers originate from cells of the TRIGEMINAL GANGLION and project to the TRIGEMINAL NUCLEUS of the brain stem. The smaller motor part arises from the brain stem trigeminal motor nucleus and innervates the muscles of mastication.
The 6th cranial nerve which originates in the ABDUCENS NUCLEUS of the PONS and sends motor fibers to the lateral rectus muscles of the EYE. Damage to the nerve or its nucleus disrupts horizontal eye movement control.
The 8th cranial nerve. The vestibulocochlear nerve has a cochlear part (COCHLEAR NERVE) which is concerned with hearing and a vestibular part (VESTIBULAR NERVE) which mediates the sense of balance and head position. The fibers of the cochlear nerve originate from neurons of the SPIRAL GANGLION and project to the cochlear nuclei (COCHLEAR NUCLEUS). The fibers of the vestibular nerve arise from neurons of Scarpa's ganglion and project to the VESTIBULAR NUCLEI.
The 2nd cranial nerve which conveys visual information from the RETINA to the brain. The nerve carries the axons of the RETINAL GANGLION CELLS which sort at the OPTIC CHIASM and continue via the OPTIC TRACTS to the brain. The largest projection is to the lateral geniculate nuclei; other targets include the SUPERIOR COLLICULI and the SUPRACHIASMATIC NUCLEI. Though known as the second cranial nerve, it is considered part of the CENTRAL NERVOUS SYSTEM.
Slender processes of NEURONS, including the AXONS and their glial envelopes (MYELIN SHEATH). Nerve fibers conduct nerve impulses to and from the CENTRAL NERVOUS SYSTEM.
A syndrome of congenital facial paralysis, frequently associated with abducens palsy and other congenital abnormalities including lingual palsy, clubfeet, brachial disorders, cognitive deficits, and pectoral muscle defects. Pathologic findings are variable and include brain stem nuclear aplasia, facial nerve aplasia, and facial muscle aplasia, consistent with a multifactorial etiology. (Adams et al., Principles of Neurology, 6th ed, p1020)
Mechanical compression of nerves or nerve roots from internal or external causes. These may result in a conduction block to nerve impulses (due to MYELIN SHEATH dysfunction) or axonal loss. The nerve and nerve sheath injuries may be caused by ISCHEMIA; INFLAMMATION; or a direct mechanical effect.
The 11th cranial nerve which originates from NEURONS in the MEDULLA and in the CERVICAL SPINAL CORD. It has a cranial root, which joins the VAGUS NERVE (10th cranial) and sends motor fibers to the muscles of the LARYNX, and a spinal root, which sends motor fibers to the TRAPEZIUS and the sternocleidomastoid muscles.
Traumatic injuries to the LARYNGEAL NERVE.
Paralysis of one or more of the ocular muscles due to disorders of the eye muscles, neuromuscular junction, supporting soft tissue, tendons, or innervation to the muscles.
Severe or complete loss of facial muscle motor function. This condition may result from central or peripheral lesions. Damage to CNS motor pathways from the cerebral cortex to the facial nuclei in the pons leads to facial weakness that generally spares the forehead muscles. FACIAL NERVE DISEASES generally results in generalized hemifacial weakness. NEUROMUSCULAR JUNCTION DISEASES and MUSCULAR DISEASES may also cause facial paralysis or paresis.
Diseases of the trigeminal nerve or its nuclei, which are located in the pons and medulla. The nerve is composed of three divisions: ophthalmic, maxillary, and mandibular, which provide sensory innervation to structures of the face, sinuses, and portions of the cranial vault. The mandibular nerve also innervates muscles of mastication. Clinical features include loss of facial and intra-oral sensation and weakness of jaw closure. Common conditions affecting the nerve include brain stem ischemia, INFRATENTORIAL NEOPLASMS, and TRIGEMINAL NEURALGIA.
Pathological processes of the VESTIBULOCOCHLEAR NERVE, including the branches of COCHLEAR NERVE and VESTIBULAR NERVE. Common examples are VESTIBULAR NEURITIS, cochlear neuritis, and ACOUSTIC NEUROMA. Clinical signs are varying degree of HEARING LOSS; VERTIGO; and TINNITUS.
Renewal or physiological repair of damaged nerve tissue.
The 4th cranial nerve. The trochlear nerve carries the motor innervation of the superior oblique muscles of the eye.
A syndrome characterized by recurrent episodes of excruciating pain lasting several seconds or longer in the sensory distribution of the TRIGEMINAL NERVE. Pain may be initiated by stimulation of trigger points on the face, lips, or gums or by movement of facial muscles or chewing. Associated conditions include MULTIPLE SCLEROSIS, vascular anomalies, ANEURYSMS, and neoplasms. (Adams et al., Principles of Neurology, 6th ed, p187)
A general term most often used to describe severe or complete loss of muscle strength due to motor system disease from the level of the cerebral cortex to the muscle fiber. This term may also occasionally refer to a loss of sensory function. (From Adams et al., Principles of Neurology, 6th ed, p45)
Diseases of the facial nerve or nuclei. Pontine disorders may affect the facial nuclei or nerve fascicle. The nerve may be involved intracranially, along its course through the petrous portion of the temporal bone, or along its extracranial course. Clinical manifestations include facial muscle weakness, loss of taste from the anterior tongue, hyperacusis, and decreased lacrimation.
Junction between the cerebellum and the pons.
Neoplasms of the base of the skull specifically, differentiated from neoplasms of unspecified sites or bones of the skull (SKULL NEOPLASMS).
Traumatic injuries to the facial nerve. This may result in FACIAL PARALYSIS, decreased lacrimation and salivation, and loss of taste sensation in the anterior tongue. The nerve may regenerate and reform its original pattern of innervation, or regenerate aberrantly, resulting in inappropriate lacrimation in response to gustatory stimuli (e.g., "crocodile tears") and other syndromes.
Traumatic injuries to the TROCHLEAR NERVE.
The 12th cranial nerve. The hypoglossal nerve originates in the hypoglossal nucleus of the medulla and supplies motor innervation to all of the muscles of the tongue except the palatoglossus (which is supplied by the vagus). This nerve also contains proprioceptive afferents from the tongue muscles.
A paraganglioma involving the glomus jugulare, a microscopic collection of chemoreceptor tissue in the adventitia of the bulb of the jugular vein. It may cause paralysis of the vocal cords, attacks of dizziness, blackouts, and nystagmus. It is not resectable but radiation therapy is effective. It regresses slowly, but permanent control is regularly achieved. (From Dorland, 27th ed; Stedman, 25th ed; DeVita Jr et al., Cancer: Principles & Practice of Oncology, 3d ed, pp1603-4)
Interruption of NEURAL CONDUCTION in peripheral nerves or nerve trunks by the injection of a local anesthetic agent (e.g., LIDOCAINE; PHENOL; BOTULINUM TOXINS) to manage or treat pain.
Branch-like terminations of NERVE FIBERS, sensory or motor NEURONS. Endings of sensory neurons are the beginnings of afferent pathway to the CENTRAL NERVOUS SYSTEM. Endings of motor neurons are the terminals of axons at the muscle cells. Nerve endings which release neurotransmitters are called PRESYNAPTIC TERMINALS.
The dense rock-like part of temporal bone that contains the INNER EAR. Petrous bone is located at the base of the skull. Sometimes it is combined with the MASTOID PROCESS and called petromastoid part of temporal bone.
A branch of the tibial nerve which supplies sensory innervation to parts of the lower leg and foot.
An irregularly shaped venous space in the dura mater at either side of the sphenoid bone.
A major nerve of the upper extremity. In humans, the fibers of the median nerve originate in the lower cervical and upper thoracic spinal cord (usually C6 to T1), travel via the brachial plexus, and supply sensory and motor innervation to parts of the forearm and hand.
Treatment of muscles and nerves under pressure as a result of crush injuries.
Injuries to the PERIPHERAL NERVES.
The medial terminal branch of the sciatic nerve. The tibial nerve fibers originate in lumbar and sacral spinal segments (L4 to S2). They supply motor and sensory innervation to parts of the calf and foot.
The inferior region of the skull consisting of an internal (cerebral), and an external (basilar) surface.
A major nerve of the upper extremity. In humans, the fibers of the ulnar nerve originate in the lower cervical and upper thoracic spinal cord (usually C7 to T1), travel via the medial cord of the brachial plexus, and supply sensory and motor innervation to parts of the hand and forearm.
A visual symptom in which a single object is perceived by the visual cortex as two objects rather than one. Disorders associated with this condition include REFRACTIVE ERRORS; STRABISMUS; OCULOMOTOR NERVE DISEASES; TROCHLEAR NERVE DISEASES; ABDUCENS NERVE DISEASES; and diseases of the BRAIN STEM and OCCIPITAL LOBE.
Diseases of the ninth cranial (glossopharyngeal) nerve or its nuclei in the medulla. The nerve may be injured by diseases affecting the lower brain stem, floor of the posterior fossa, jugular foramen, or the nerve's extracranial course. Clinical manifestations include loss of sensation from the pharynx, decreased salivation, and syncope. Glossopharyngeal neuralgia refers to a condition that features recurrent unilateral sharp pain in the tongue, angle of the jaw, external auditory meatus and throat that may be associated with SYNCOPE. Episodes may be triggered by cough, sneeze, swallowing, or pressure on the tragus of the ear. (Adams et al., Principles of Neurology, 6th ed, p1390)
The infratentorial compartment that contains the CEREBELLUM and BRAIN STEM. It is formed by the posterior third of the superior surface of the body of the sphenoid (SPHENOID BONE), by the occipital, the petrous, and mastoid portions of the TEMPORAL BONE, and the posterior inferior angle of the PARIETAL BONE.
A nerve originating in the lumbar spinal cord (usually L2 to L4) and traveling through the lumbar plexus to provide motor innervation to extensors of the thigh and sensory innervation to parts of the thigh, lower leg, and foot, and to the hip and knee joints.
Non-invasive method of demonstrating internal anatomy based on the principle that atomic nuclei in a strong magnetic field absorb pulses of radiofrequency energy and emit them as radiowaves which can be reconstructed into computerized images. The concept includes proton spin tomographic techniques.
The 31 paired peripheral nerves formed by the union of the dorsal and ventral spinal roots from each spinal cord segment. The spinal nerve plexuses and the spinal roots are also included.
The cochlear part of the 8th cranial nerve (VESTIBULOCOCHLEAR NERVE). The cochlear nerve fibers originate from neurons of the SPIRAL GANGLION and project peripherally to cochlear hair cells and centrally to the cochlear nuclei (COCHLEAR NUCLEUS) of the BRAIN STEM. They mediate the sense of hearing.
A neoplasm that arises from SCHWANN CELLS of the cranial, peripheral, and autonomic nerves. Clinically, these tumors may present as a cranial neuropathy, abdominal or soft tissue mass, intracranial lesion, or with spinal cord compression. Histologically, these tumors are encapsulated, highly vascular, and composed of a homogenous pattern of biphasic fusiform-shaped cells that may have a palisaded appearance. (From DeVita Jr et al., Cancer: Principles and Practice of Oncology, 5th ed, pp964-5)
Traumatic injuries to the GLOSSOPHARYNGEAL NERVE.
Radiography of the central nervous system.
NERVE GROWTH FACTOR is the first of a series of neurotrophic factors that were found to influence the growth and differentiation of sympathetic and sensory neurons. It is comprised of alpha, beta, and gamma subunits. The beta subunit is responsible for its growth stimulating activity.
Factors which enhance the growth potentialities of sensory and sympathetic nerve cells.
Muscles of facial expression or mimetic muscles that include the numerous muscles supplied by the facial nerve that are attached to and move the skin of the face. (From Stedman, 25th ed)
A benign SCHWANNOMA of the eighth cranial nerve (VESTIBULOCOCHLEAR NERVE), mostly arising from the vestibular branch (VESTIBULAR NERVE) during the fifth or sixth decade of life. Clinical manifestations include HEARING LOSS; HEADACHE; VERTIGO; TINNITUS; and FACIAL PAIN. Bilateral acoustic neuromas are associated with NEUROFIBROMATOSIS 2. (From Adams et al., Principles of Neurology, 6th ed, p673)
Traumatic injuries to the VAGUS NERVE. Because the vagus nerve innervates multiple organs, injuries in the nerve fibers may result in any gastrointestinal organ dysfunction downstream of the injury site.
Benign paraganglioma at the bifurcation of the COMMON CAROTID ARTERIES. It can encroach on the parapharyngeal space and produce dysphagia, pain, and cranial nerve palsies.
The motor nerve of the diaphragm. The phrenic nerve fibers originate in the cervical spinal column (mostly C4) and travel through the cervical plexus to the diaphragm.
A syndrome characterized by marked limitation of abduction of the eye, variable limitation of adduction and retraction of the globe, and narrowing of the palpebral fissure on attempted adduction. The condition is caused by aberrant innervation of the lateral rectus by fibers of the OCULOMOTOR NERVE.
A major nerve of the upper extremity. In humans the fibers of the radial nerve originate in the lower cervical and upper thoracic spinal cord (usually C5 to T1), travel via the posterior cord of the brachial plexus, and supply motor innervation to extensor muscles of the arm and cutaneous sensory fibers to extensor regions of the arm and hand.
Branches of the vagus (tenth cranial) nerve. The recurrent laryngeal nerves originate more caudally than the superior laryngeal nerves and follow different paths on the right and left sides. They carry efferents to all muscles of the larynx except the cricothyroid and carry sensory and autonomic fibers to the laryngeal, pharyngeal, tracheal, and cardiac regions.
The part of the brain that connects the CEREBRAL HEMISPHERES with the SPINAL CORD. It consists of the MESENCEPHALON; PONS; and MEDULLA OBLONGATA.
Paired bundles of NERVE FIBERS entering and leaving the SPINAL CORD at each segment. The dorsal and ventral nerve roots join to form the mixed segmental spinal nerves. The dorsal roots are generally afferent, formed by the central projections of the spinal (dorsal root) ganglia sensory cells, and the ventral roots are efferent, comprising the axons of spinal motor and PREGANGLIONIC AUTONOMIC FIBERS.
Primary or secondary neoplasm in the ARACHNOID or SUBARACHNOID SPACE. It appears as a diffuse fibrotic thickening of the MENINGES associated with variable degrees of inflammation.
Diseases of the twelfth cranial (hypoglossal) nerve or nuclei. The nuclei and fascicles of the nerve are located in the medulla, and the nerve exits the skull via the hypoglossal foramen and innervates the muscles of the tongue. Lower brain stem diseases, including ischemia and MOTOR NEURON DISEASES may affect the nuclei or nerve fascicles. The nerve may also be injured by diseases of the posterior fossa or skull base. Clinical manifestations include unilateral weakness of tongue musculature and lingual dysarthria, with deviation of the tongue towards the side of weakness upon attempted protrusion.
Increase in the mass of bone per unit volume.
The 1st cranial nerve. The olfactory nerve conveys the sense of smell. It is formed by the axons of OLFACTORY RECEPTOR NEURONS which project from the olfactory epithelium (in the nasal epithelium) to the OLFACTORY BULB.
A sensory branch of the trigeminal (5th cranial) nerve. The ophthalmic nerve carries general afferents from the superficial division of the face including the eyeball, conjunctiva, upper eyelid, upper nose, nasal mucosa, and scalp.
The propagation of the NERVE IMPULSE along the nerve away from the site of an excitation stimulus.
Either of a pair of compound bones forming the lateral (left and right) surfaces and base of the skull which contains the organs of hearing. It is a large bone formed by the fusion of parts: the squamous (the flattened anterior-superior part), the tympanic (the curved anterior-inferior part), the mastoid (the irregular posterior portion), and the petrous (the part at the base of the skull).
A syndrome characterized by headache, neck stiffness, low grade fever, and CSF lymphocytic pleocytosis in the absence of an acute bacterial pathogen. Viral meningitis is the most frequent cause although MYCOPLASMA INFECTIONS; RICKETTSIA INFECTIONS; diagnostic or therapeutic procedures; NEOPLASTIC PROCESSES; septic perimeningeal foci; and other conditions may result in this syndrome. (From Adams et al., Principles of Neurology, 6th ed, p745)
Differentiated tissue of the central nervous system composed of NERVE CELLS, fibers, DENDRITES, and specialized supporting cells.
A branch of the trigeminal (5th cranial) nerve. The mandibular nerve carries motor fibers to the muscles of mastication and sensory fibers to the teeth and gingivae, the face in the region of the mandible, and parts of the dura.
Traumatic injuries to the RECURRENT LARYNGEAL NERVE that may result in vocal cord dysfunction.
Recurrent clonic contraction of facial muscles, restricted to one side. It may occur as a manifestation of compressive lesions involving the seventh cranial nerve (FACIAL NERVE DISEASES), during recovery from BELL PALSY, or in association with other disorders. (From Adams et al., Principles of Neurology, 6th ed, p1378)
Games in which players use a racquet to hit a ball or similar type object.
Neoplasms of the bony part of the skull.
Nerve fibers that are capable of rapidly conducting impulses away from the neuron cell body.
The muscles that move the eye. Included in this group are the medial rectus, lateral rectus, superior rectus, inferior rectus, inferior oblique, superior oblique, musculus orbitalis, and levator palpebrae superioris.
Traumatic injury to the abducens, or sixth, cranial nerve. Injury to this nerve results in lateral rectus muscle weakness or paralysis. The nerve may be damaged by closed or penetrating CRANIOCEREBRAL TRAUMA or by facial trauma involving the orbit.
A relatively common neoplasm of the CENTRAL NERVOUS SYSTEM that arises from arachnoidal cells. The majority are well differentiated vascular tumors which grow slowly and have a low potential to be invasive, although malignant subtypes occur. Meningiomas have a predilection to arise from the parasagittal region, cerebral convexity, sphenoidal ridge, olfactory groove, and SPINAL CANAL. (From DeVita et al., Cancer: Principles and Practice of Oncology, 5th ed, pp2056-7)
The large hole at the base of the skull through which the SPINAL CORD passes.
Part of the back and base of the CRANIUM that encloses the FORAMEN MAGNUM.
One of the paired air spaces located in the body of the SPHENOID BONE behind the ETHMOID BONE in the middle of the skull. Sphenoid sinus communicates with the posterosuperior part of NASAL CAVITY on the same side.
The posterior of the three primitive cerebral vesicles of an embryonic brain. It consists of myelencephalon, metencephalon, and isthmus rhombencephali from which develop the major BRAIN STEM components, such as MEDULLA OBLONGATA from the myelencephalon, CEREBELLUM and PONS from the metencephalon, with the expanded cavity forming the FOURTH VENTRICLE.
An involuntary contraction of a muscle or group of muscles. Spasms may involve SKELETAL MUSCLE or SMOOTH MUSCLE.
The major nerves supplying sympathetic innervation to the abdomen. The greater, lesser, and lowest (or smallest) splanchnic nerves are formed by preganglionic fibers from the spinal cord which pass through the paravertebral ganglia and then to the celiac ganglia and plexuses. The lumbar splanchnic nerves carry fibers which pass through the lumbar paravertebral ganglia to the mesenteric and hypogastric ganglia.
A muscular organ in the mouth that is covered with pink tissue called mucosa, tiny bumps called papillae, and thousands of taste buds. The tongue is anchored to the mouth and is vital for chewing, swallowing, and for speech.
An illusion of movement, either of the external world revolving around the individual or of the individual revolving in space. Vertigo may be associated with disorders of the inner ear (EAR, INNER); VESTIBULAR NERVE; BRAINSTEM; or CEREBRAL CORTEX. Lesions in the TEMPORAL LOBE and PARIETAL LOBE may be associated with FOCAL SEIZURES that may feature vertigo as an ictal manifestation. (From Adams et al., Principles of Neurology, 6th ed, pp300-1)
Benign and malignant neoplastic processes that arise from or secondarily involve the meningeal coverings of the brain and spinal cord.
Assessment of sensory and motor responses and reflexes that is used to determine impairment of the nervous system.
The performance of surgical procedures with the aid of a microscope.
Injuries to the optic nerve induced by a trauma to the face or head. These may occur with closed or penetrating injuries. Relatively minor compression of the superior aspect of orbit may also result in trauma to the optic nerve. Clinical manifestations may include visual loss, PAPILLEDEMA, and an afferent pupillary defect.
Diseases characterized by injury or dysfunction involving multiple peripheral nerves and nerve roots. The process may primarily affect myelin or nerve axons. Two of the more common demyelinating forms are acute inflammatory polyradiculopathy (GUILLAIN-BARRE SYNDROME) and POLYRADICULONEUROPATHY, CHRONIC INFLAMMATORY DEMYELINATING. Polyradiculoneuritis refers to inflammation of multiple peripheral nerves and spinal nerve roots.
A syndrome associated with defective sympathetic innervation to one side of the face, including the eye. Clinical features include MIOSIS; mild BLEPHAROPTOSIS; and hemifacial ANHIDROSIS (decreased sweating)(see HYPOHIDROSIS). Lesions of the BRAIN STEM; cervical SPINAL CORD; first thoracic nerve root; apex of the LUNG; CAROTID ARTERY; CAVERNOUS SINUS; and apex of the ORBIT may cause this condition. (From Miller et al., Clinical Neuro-Ophthalmology, 4th ed, pp500-11)
Conditions which produce injury or dysfunction of the second cranial or optic nerve, which is generally considered a component of the central nervous system. Damage to optic nerve fibers may occur at or near their origin in the retina, at the optic disk, or in the nerve, optic chiasm, optic tract, or lateral geniculate nuclei. Clinical manifestations may include decreased visual acuity and contrast sensitivity, impaired color vision, and an afferent pupillary defect.
Any operation on the cranium or incision into the cranium. (Dorland, 28th ed)
Branch of the common carotid artery which supplies the anterior part of the brain, the eye and its appendages, the forehead and nose.
The three membranes that cover the BRAIN and the SPINAL CORD. They are the dura mater, the arachnoid, and the pia mater.
A syndrome characterized by facial palsy in association with a herpetic eruption of the external auditory meatus. This may occasionally be associated with tinnitus, vertigo, deafness, severe otalgia, and inflammation of the pinna. The condition is caused by reactivation of a latent HERPESVIRUS 3, HUMAN infection which causes inflammation of the facial and vestibular nerves, and may occasionally involve additional cranial nerves. (From Adams et al., Principles of Neurology, 6th ed, p757)
The twelve spinal nerves on each side of the thorax. They include eleven INTERCOSTAL NERVES and one subcostal nerve. Both sensory and motor, they supply the muscles and skin of the thoracic and abdominal walls.
A class of nerve fibers as defined by their structure, specifically the nerve sheath arrangement. The AXONS of the myelinated nerve fibers are completely encased in a MYELIN SHEATH. They are fibers of relatively large and varied diameters. Their NEURAL CONDUCTION rates are faster than those of the unmyelinated nerve fibers (NERVE FIBERS, UNMYELINATED). Myelinated nerve fibers are present in somatic and autonomic nerves.
An autosomal dominant disorder characterized by a high incidence of bilateral acoustic neuromas as well as schwannomas (NEURILEMMOMA) of other cranial and peripheral nerves, and other benign intracranial tumors including meningiomas, ependymomas, spinal neurofibromas, and gliomas. The disease has been linked to mutations of the NF2 gene (GENES, NEUROFIBROMATOSIS 2) on chromosome 22 (22q12) and usually presents clinically in the first or second decade of life.
Neurons which activate MUSCLE CELLS.
Surgery performed on the nervous system or its parts.
Benign and malignant intra-axial tumors of the MESENCEPHALON; PONS; or MEDULLA OBLONGATA of the BRAIN STEM. Primary and metastatic neoplasms may occur in this location. Clinical features include ATAXIA, cranial neuropathies (see CRANIAL NERVE DISEASES), NAUSEA, hemiparesis (see HEMIPLEGIA), and quadriparesis. Primary brain stem neoplasms are more frequent in children. Histologic subtypes include GLIOMA; HEMANGIOBLASTOMA; GANGLIOGLIOMA; and EPENDYMOMA.
Branches of the VAGUS NERVE. The superior laryngeal nerves originate near the nodose ganglion and separate into external branches, which supply motor fibers to the cricothyroid muscles, and internal branches, which carry sensory fibers. The RECURRENT LARYNGEAL NERVE originates more caudally and carries efferents to all muscles of the larynx except the cricothyroid. The laryngeal nerves and their various branches also carry sensory and autonomic fibers to the laryngeal, pharyngeal, tracheal, and cardiac regions.
The excision of the thickened, atheromatous tunica intima of a carotid artery.
Intracranial tumors originating in the region of the brain inferior to the tentorium cerebelli, which contains the cerebellum, fourth ventricle, cerebellopontine angle, brain stem, and related structures. Primary tumors of this region are more frequent in children, and may present with ATAXIA; CRANIAL NERVE DISEASES; vomiting; HEADACHE; HYDROCEPHALUS; or other signs of neurologic dysfunction. Relatively frequent histologic subtypes include TERATOMA; MEDULLOBLASTOMA; GLIOBLASTOMA; ASTROCYTOMA; EPENDYMOMA; CRANIOPHARYNGIOMA; and choroid plexus papilloma (PAPILLOMA, CHOROID PLEXUS).
The thoracolumbar division of the autonomic nervous system. Sympathetic preganglionic fibers originate in neurons of the intermediolateral column of the spinal cord and project to the paravertebral and prevertebral ganglia, which in turn project to target organs. The sympathetic nervous system mediates the body's response to stressful situations, i.e., the fight or flight reactions. It often acts reciprocally to the parasympathetic system.
The space between the arachnoid membrane and PIA MATER, filled with CEREBROSPINAL FLUID. It contains large blood vessels that supply the BRAIN and SPINAL CORD.
Swelling of the OPTIC DISK, usually in association with increased intracranial pressure, characterized by hyperemia, blurring of the disk margins, microhemorrhages, blind spot enlargement, and engorgement of retinal veins. Chronic papilledema may cause OPTIC ATROPHY and visual loss. (Miller et al., Clinical Neuro-Ophthalmology, 4th ed, p175)
Neurons which conduct NERVE IMPULSES to the CENTRAL NERVOUS SYSTEM.
Tomography using x-ray transmission and a computer algorithm to reconstruct the image.
An acute inflammatory autoimmune neuritis caused by T cell- mediated cellular immune response directed towards peripheral myelin. Demyelination occurs in peripheral nerves and nerve roots. The process is often preceded by a viral or bacterial infection, surgery, immunization, lymphoma, or exposure to toxins. Common clinical manifestations include progressive weakness, loss of sensation, and loss of deep tendon reflexes. Weakness of respiratory muscles and autonomic dysfunction may occur. (From Adams et al., Principles of Neurology, 6th ed, pp1312-1314)
An irregular unpaired bone situated at the SKULL BASE and wedged between the frontal, temporal, and occipital bones (FRONTAL BONE; TEMPORAL BONE; OCCIPITAL BONE). Sphenoid bone consists of a median body and three pairs of processes resembling a bat with spread wings. The body is hollowed out in its inferior to form two large cavities (SPHENOID SINUS).
Peripheral, autonomic, and cranial nerve disorders that are associated with DIABETES MELLITUS. These conditions usually result from diabetic microvascular injury involving small blood vessels that supply nerves (VASA NERVORUM). Relatively common conditions which may be associated with diabetic neuropathy include third nerve palsy (see OCULOMOTOR NERVE DISEASES); MONONEUROPATHY; mononeuropathy multiplex; diabetic amyotrophy; a painful POLYNEUROPATHY; autonomic neuropathy; and thoracoabdominal neuropathy. (From Adams et al., Principles of Neurology, 6th ed, p1325)
The outermost of the three MENINGES, a fibrous membrane of connective tissue that covers the brain and the spinal cord.
Use of electric potential or currents to elicit biological responses.
A region, of SOMITE development period, that contains a number of paired arches, each with a mesodermal core lined by ectoderm and endoderm on the two sides. In lower aquatic vertebrates, branchial arches develop into GILLS. In higher vertebrates, the arches forms outpouchings and develop into structures of the head and neck. Separating the arches are the branchial clefts or grooves.
A sensory branch of the MANDIBULAR NERVE, which is part of the trigeminal (5th cranial) nerve. The lingual nerve carries general afferent fibers from the anterior two-thirds of the tongue, the floor of the mouth, and the mandibular gingivae.
The posterior part of the temporal bone. It is a projection of the petrous bone.
The front part of the hindbrain (RHOMBENCEPHALON) that lies between the MEDULLA and the midbrain (MESENCEPHALON) ventral to the cerebellum. It is composed of two parts, the dorsal and the ventral. The pons serves as a relay station for neural pathways between the CEREBELLUM to the CEREBRUM.
Clinical signs and symptoms caused by nervous system injury or dysfunction.
An abnormal response to a stimulus applied to the sensory components of the nervous system. This may take the form of increased, decreased, or absent reflexes.
A motor neuron disease marked by progressive weakness of the muscles innervated by cranial nerves of the lower brain stem. Clinical manifestations include dysarthria, dysphagia, facial weakness, tongue weakness, and fasciculations of the tongue and facial muscles. The adult form of the disease is marked initially by bulbar weakness which progresses to involve motor neurons throughout the neuroaxis. Eventually this condition may become indistinguishable from AMYOTROPHIC LATERAL SCLEROSIS. Fazio-Londe syndrome is an inherited form of this illness which occurs in children and young adults. (Adams et al., Principles of Neurology, 6th ed, p1091; Brain 1992 Dec;115(Pt 6):1889-1900)
Abnormal outpouching in the wall of intracranial blood vessels. Most common are the saccular (berry) aneurysms located at branch points in CIRCLE OF WILLIS at the base of the brain. Vessel rupture results in SUBARACHNOID HEMORRHAGE or INTRACRANIAL HEMORRHAGES. Giant aneurysms (>2.5 cm in diameter) may compress adjacent structures, including the OCULOMOTOR NERVE. (From Adams et al., Principles of Neurology, 6th ed, p841)
A form of bacterial meningitis caused by MYCOBACTERIUM TUBERCULOSIS or rarely MYCOBACTERIUM BOVIS. The organism seeds the meninges and forms microtuberculomas which subsequently rupture. The clinical course tends to be subacute, with progressions occurring over a period of several days or longer. Headache and meningeal irritation may be followed by SEIZURES, cranial neuropathies, focal neurologic deficits, somnolence, and eventually COMA. The illness may occur in immunocompetent individuals or as an OPPORTUNISTIC INFECTION in the ACQUIRED IMMUNODEFICIENCY SYNDROME and other immunodeficiency syndromes. (From Adams et al., Principles of Neurology, 6th ed, pp717-9)
Studies used to test etiologic hypotheses in which inferences about an exposure to putative causal factors are derived from data relating to characteristics of persons under study or to events or experiences in their past. The essential feature is that some of the persons under study have the disease or outcome of interest and their characteristics are compared with those of unaffected persons.
Antiserum given therapeutically in BOTULISM.
Death resulting from the presence of a disease in an individual, as shown by a single case report or a limited number of patients. This should be differentiated from DEATH, the physiological cessation of life and from MORTALITY, an epidemiological or statistical concept.
Loss of functional activity and trophic degeneration of nerve axons and their terminal arborizations following the destruction of their cells of origin or interruption of their continuity with these cells. The pathology is characteristic of neurodegenerative diseases. Often the process of nerve degeneration is studied in research on neuroanatomical localization and correlation of the neurophysiology of neural pathways.
A developmental deformity of the occipital bone and upper end of the cervical spine, in which the latter appears to have pushed the floor of the occipital bone upward. (Dorland, 27th ed)
Formation or presence of a blood clot (THROMBUS) in the SUPERIOR SAGITTAL SINUS or the inferior sagittal sinus. Sagittal sinus thrombosis can result from infections, hematological disorders, CRANIOCEREBRAL TRAUMA; and NEUROSURGICAL PROCEDURES. Clinical features are primarily related to the increased intracranial pressure causing HEADACHE; NAUSEA; and VOMITING. Severe cases can evolve to SEIZURES or COMA.
A cylindrical column of tissue that lies within the vertebral canal. It is composed of WHITE MATTER and GRAY MATTER.
Inflammation of the OUTER EAR including the external EAR CANAL, cartilages of the auricle (EAR CARTILAGE), and the TYMPANIC MEMBRANE.
A nonspecific symptom of hearing disorder characterized by the sensation of buzzing, ringing, clicking, pulsations, and other noises in the ear. Objective tinnitus refers to noises generated from within the ear or adjacent structures that can be heard by other individuals. The term subjective tinnitus is used when the sound is audible only to the affected individual. Tinnitus may occur as a manifestation of COCHLEAR DISEASES; VESTIBULOCOCHLEAR NERVE DISEASES; INTRACRANIAL HYPERTENSION; CRANIOCEREBRAL TRAUMA; and other conditions.
A method of hemostasis utilizing various agents such as Gelfoam, silastic, metal, glass, or plastic pellets, autologous clot, fat, and muscle as emboli. It has been used in the treatment of spinal cord and INTRACRANIAL ARTERIOVENOUS MALFORMATIONS, renal arteriovenous fistulas, gastrointestinal bleeding, epistaxis, hypersplenism, certain highly vascular tumors, traumatic rupture of blood vessels, and control of operative hemorrhage.
The hearing and equilibrium system of the body. It consists of three parts: the EXTERNAL EAR, the MIDDLE EAR, and the INNER EAR. Sound waves are transmitted through this organ where vibration is transduced to nerve signals that pass through the ACOUSTIC NERVE to the CENTRAL NERVOUS SYSTEM. The inner ear also contains the vestibular organ that maintains equilibrium by transducing signals to the VESTIBULAR NERVE.
A disease caused by potent protein NEUROTOXINS produced by CLOSTRIDIUM BOTULINUM which interfere with the presynaptic release of ACETYLCHOLINE at the NEUROMUSCULAR JUNCTION. Clinical features include abdominal pain, vomiting, acute PARALYSIS (including respiratory paralysis), blurred vision, and DIPLOPIA. Botulism may be classified into several subtypes (e.g., food-borne, infant, wound, and others). (From Adams et al., Principles of Neurology, 6th ed, p1208)
Congenital or acquired paralysis of one or both VOCAL CORDS. This condition is caused by defects in the CENTRAL NERVOUS SYSTEM, the VAGUS NERVE and branches of LARYNGEAL NERVES. Common symptoms are VOICE DISORDERS including HOARSENESS or APHONIA.
The 10th cranial nerve. The vagus is a mixed nerve which contains somatic afferents (from skin in back of the ear and the external auditory meatus), visceral afferents (from the pharynx, larynx, thorax, and abdomen), parasympathetic efferents (to the thorax and abdomen), and efferents to striated muscle (of the larynx and pharynx).
Cells specialized to detect chemical substances and relay that information centrally in the nervous system. Chemoreceptor cells may monitor external stimuli, as in TASTE and OLFACTION, or internal stimuli, such as the concentrations of OXYGEN and CARBON DIOXIDE in the blood.
A radiological stereotactic technique developed for cutting or destroying tissue by high doses of radiation in place of surgical incisions. It was originally developed for neurosurgery on structures in the brain and its use gradually spread to radiation surgery on extracranial structures as well. The usual rigid needles or probes of stereotactic surgery are replaced with beams of ionizing radiation directed toward a target so as to achieve local tissue destruction.
Diseases that affect the structure or function of the cerebellum. Cardinal manifestations of cerebellar dysfunction include dysmetria, GAIT ATAXIA, and MUSCLE HYPOTONIA.
Bony cavity that holds the eyeball and its associated tissues and appendages.
A variant of the GUILLAIN-BARRE SYNDROME characterized by the acute onset of oculomotor dysfunction, ataxia, and loss of deep tendon reflexes with relative sparing of strength in the extremities and trunk. The ataxia is produced by peripheral sensory nerve dysfunction and not by cerebellar injury. Facial weakness and sensory loss may also occur. The process is mediated by autoantibodies directed against a component of myelin found in peripheral nerves. (Adams et al., Principles of Neurology, 6th ed, p1313; Neurology 1987 Sep;37(9):1493-8)
The domestic cat, Felis catus, of the carnivore family FELIDAE, comprising over 30 different breeds. The domestic cat is descended primarily from the wild cat of Africa and extreme southwestern Asia. Though probably present in towns in Palestine as long ago as 7000 years, actual domestication occurred in Egypt about 4000 years ago. (From Walker's Mammals of the World, 6th ed, p801)
The lower portion of the BRAIN STEM. It is inferior to the PONS and anterior to the CEREBELLUM. Medulla oblongata serves as a relay station between the brain and the spinal cord, and contains centers for regulating respiratory, vasomotor, cardiac, and reflex activities.
Evaluation undertaken to assess the results or consequences of management and procedures used in combating disease in order to determine the efficacy, effectiveness, safety, and practicability of these interventions in individual cases or series.
The intermediate sensory division of the trigeminal (5th cranial) nerve. The maxillary nerve carries general afferents from the intermediate region of the face including the lower eyelid, nose and upper lip, the maxillary teeth, and parts of the dura.
The two longitudinal ridges along the PRIMITIVE STREAK appearing near the end of GASTRULATION during development of nervous system (NEURULATION). The ridges are formed by folding of NEURAL PLATE. Between the ridges is a neural groove which deepens as the fold become elevated. When the folds meet at midline, the groove becomes a closed tube, the NEURAL TUBE.
Clusters of multipolar neurons surrounded by a capsule of loosely organized CONNECTIVE TISSUE located outside the CENTRAL NERVOUS SYSTEM.
The SKELETON of the HEAD including the FACIAL BONES and the bones enclosing the BRAIN.
Disorders of speech articulation caused by imperfect coordination of pharynx, larynx, tongue, or face muscles. This may result from CRANIAL NERVE DISEASES; NEUROMUSCULAR DISEASES; CEREBELLAR DISEASES; BASAL GANGLIA DISEASES; BRAIN STEM diseases; or diseases of the corticobulbar tracts (see PYRAMIDAL TRACTS). The cortical language centers are intact in this condition. (From Adams et al., Principles of Neurology, 6th ed, p489)
Neoplasms which arise from nerve sheaths formed by SCHWANN CELLS in the PERIPHERAL NERVOUS SYSTEM or by OLIGODENDROCYTES in the CENTRAL NERVOUS SYSTEM. Malignant peripheral nerve sheath tumors, NEUROFIBROMA, and NEURILEMMOMA are relatively common tumors in this category.
Diseases of the peripheral nerves external to the brain and spinal cord, which includes diseases of the nerve roots, ganglia, plexi, autonomic nerves, sensory nerves, and motor nerves.
Cell surface receptors that bind NERVE GROWTH FACTOR; (NGF) and a NGF-related family of neurotrophic factors that includes neurotrophins, BRAIN-DERIVED NEUROTROPHIC FACTOR and CILIARY NEUROTROPHIC FACTOR.
A syndrome characterized by the acute onset of unilateral FACIAL PARALYSIS which progresses over a 2-5 day period. Weakness of the orbicularis oculi muscle and resulting incomplete eye closure may be associated with corneal injury. Pain behind the ear often precedes the onset of paralysis. This condition may be associated with HERPESVIRUS 1, HUMAN infection of the facial nerve. (Adams et al., Principles of Neurology, 6th ed, p1376)
Pathologic conditions affecting the BRAIN, which is composed of the intracranial components of the CENTRAL NERVOUS SYSTEM. This includes (but is not limited to) the CEREBRAL CORTEX; intracranial white matter; BASAL GANGLIA; THALAMUS; HYPOTHALAMUS; BRAIN STEM; and CEREBELLUM.
A surgical operation for the relief of pressure in a body compartment or on a body part. (From Dorland, 28th ed)
Inflammation of the coverings of the brain and/or spinal cord, which consist of the PIA MATER; ARACHNOID; and DURA MATER. Infections (viral, bacterial, and fungal) are the most common causes of this condition, but subarachnoid hemorrhage (HEMORRHAGES, SUBARACHNOID), chemical irritation (chemical MENINGITIS), granulomatous conditions, neoplastic conditions (CARCINOMATOUS MENINGITIS), and other inflammatory conditions may produce this syndrome. (From Joynt, Clinical Neurology, 1994, Ch24, p6)
Small sensory organs which contain gustatory receptor cells, basal cells, and supporting cells. Taste buds in humans are found in the epithelia of the tongue, palate, and pharynx. They are innervated by the CHORDA TYMPANI NERVE (a branch of the facial nerve) and the GLOSSOPHARYNGEAL NERVE.
Branch of the common carotid artery which supplies the exterior of the head, the face, and the greater part of the neck.
Infarctions that occur in the BRAIN STEM which is comprised of the MIDBRAIN; PONS; and MEDULLA OBLONGATA. There are several named syndromes characterized by their distinctive clinical manifestations and specific sites of ischemic injury.
Diseases of any component of the brain (including the cerebral hemispheres, diencephalon, brain stem, and cerebellum) or the spinal cord.
The part of CENTRAL NERVOUS SYSTEM that is contained within the skull (CRANIUM). Arising from the NEURAL TUBE, the embryonic brain is comprised of three major parts including PROSENCEPHALON (the forebrain); MESENCEPHALON (the midbrain); and RHOMBENCEPHALON (the hindbrain). The developed brain consists of CEREBRUM; CEREBELLUM; and other structures in the BRAIN STEM.
Each of the upper and lower folds of SKIN which cover the EYE when closed.
The resection or removal of the nerve to an organ or part. (Dorland, 28th ed)
Disease or damage involving the SCIATIC NERVE, which divides into the PERONEAL NERVE and TIBIAL NERVE (see also PERONEAL NEUROPATHIES and TIBIAL NEUROPATHY). Clinical manifestations may include SCIATICA or pain localized to the hip, PARESIS or PARALYSIS of posterior thigh muscles and muscles innervated by the peroneal and tibial nerves, and sensory loss involving the lateral and posterior thigh, posterior and lateral leg, and sole of the foot. The sciatic nerve may be affected by trauma; ISCHEMIA; COLLAGEN DISEASES; and other conditions. (From Adams et al., Principles of Neurology, 6th ed, p1363)
The sudden loss of blood supply to the PITUITARY GLAND, leading to tissue NECROSIS and loss of function (PANHYPOPITUITARISM). The most common cause is hemorrhage or INFARCTION of a PITUITARY ADENOMA. It can also result from acute hemorrhage into SELLA TURCICA due to HEAD TRAUMA; INTRACRANIAL HYPERTENSION; or other acute effects of central nervous system hemorrhage. Clinical signs include severe HEADACHE; HYPOTENSION; bilateral visual disturbances; UNCONSCIOUSNESS; and COMA.
Diseases affecting the eye.
Electrical waves in the CEREBRAL CORTEX generated by BRAIN STEM structures in response to auditory click stimuli. These are found to be abnormal in many patients with CEREBELLOPONTINE ANGLE lesions, MULTIPLE SCLEROSIS, or other DEMYELINATING DISEASES.
A characteristic symptom complex.
Intradermal or subcutaneous saclike structure, the wall of which is stratified epithelium containing keratohyalin granules.
Large endothelium-lined venous channels situated between the two layers of DURA MATER, the endosteal and the meningeal layers. They are devoid of valves and are parts of the venous system of dura mater. Major cranial sinuses include a postero-superior group (such as superior sagittal, inferior sagittal, straight, transverse, and occipital) and an antero-inferior group (such as cavernous, petrosal, and basilar plexus).
The entire nerve apparatus, composed of a central part, the brain and spinal cord, and a peripheral part, the cranial and spinal nerves, autonomic ganglia, and plexuses. (Stedman, 26th ed)
A branch of the facial (7th cranial) nerve which passes through the middle ear and continues through the petrotympanic fissure. The chorda tympani nerve carries taste sensation from the anterior two-thirds of the tongue and conveys parasympathetic efferents to the salivary glands.
Misalignment of the visual axes of the eyes. In comitant strabismus the degree of ocular misalignment does not vary with the direction of gaze. In noncomitant strabismus the degree of misalignment varies depending on direction of gaze or which eye is fixating on the target. (Miller, Walsh & Hoyt's Clinical Neuro-Ophthalmology, 4th ed, p641)
Pathological conditions involving the CAROTID ARTERIES, including the common, internal, and external carotid arteries. ATHEROSCLEROSIS and TRAUMA are relatively frequent causes of carotid artery pathology.
An idiopathic systemic inflammatory granulomatous disorder comprised of epithelioid and multinucleated giant cells with little necrosis. It usually invades the lungs with fibrosis and may also involve lymph nodes, skin, liver, spleen, eyes, phalangeal bones, and parotid glands.
Paired respiratory organs of fishes and some amphibians that are analogous to lungs. They are richly supplied with blood vessels by which oxygen and carbon dioxide are exchanged directly with the environment.
Studies in which individuals or populations are followed to assess the outcome of exposures, procedures, or effects of a characteristic, e.g., occurrence of disease.
The vestibular part of the 8th cranial nerve (VESTIBULOCOCHLEAR NERVE). The vestibular nerve fibers arise from neurons of Scarpa's ganglion and project peripherally to vestibular hair cells and centrally to the VESTIBULAR NUCLEI of the BRAIN STEM. These fibers mediate the sense of balance and head position.
Fractures of the skull which may result from penetrating or nonpenetrating head injuries or rarely BONE DISEASES (see also FRACTURES, SPONTANEOUS). Skull fractures may be classified by location (e.g., SKULL FRACTURE, BASILAR), radiographic appearance (e.g., linear), or based upon cranial integrity (e.g., SKULL FRACTURE, DEPRESSED).
Histochemical localization of immunoreactive substances using labeled antibodies as reagents.
A class of nerve fibers as defined by their nerve sheath arrangement. The AXONS of the unmyelinated nerve fibers are small in diameter and usually several are surrounded by a single MYELIN SHEATH. They conduct low-velocity impulses, and represent the majority of peripheral sensory and autonomic fibers, but are also found in the BRAIN and SPINAL CORD.
Recording of the changes in electric potential of muscle by means of surface or needle electrodes.
Pathologic processes that affect patients after a surgical procedure. They may or may not be related to the disease for which the surgery was done, and they may or may not be direct results of the surgery.
The part of a human or animal body connecting the HEAD to the rest of the body.
A region extending from the PONS & MEDULLA OBLONGATA through the MESENCEPHALON, characterized by a diversity of neurons of various sizes and shapes, arranged in different aggregations and enmeshed in a complicated fiber network.
Tumors or cancer of the NASOPHARYNX.
The main information-processing organs of the nervous system, consisting of the brain, spinal cord, and meninges.
Narrowing or stricture of any part of the CAROTID ARTERIES, most often due to atherosclerotic plaque formation. Ulcerations may form in atherosclerotic plaques and induce THROMBUS formation. Platelet or cholesterol emboli may arise from stenotic carotid lesions and induce a TRANSIENT ISCHEMIC ATTACK; CEREBROVASCULAR ACCIDENT; or temporary blindness (AMAUROSIS FUGAX). (From Adams et al., Principles of Neurology, 6th ed, pp 822-3)

Differential distribution of retinoic acid synthesis in the chicken embryo as determined by immunolocalization of the retinoic acid synthetic enzyme, RALDH-2. (1/248)

Retinaldehyde dehydrogenase type 2 (RALDH-2) is a major retinoic acid generating enzyme in the early embryo. Here we report the immunolocalization of this enzyme (RALDH-2-IR) in stage 6-29 chicken embryos; we also show that tissues that exhibit strong RALDH-2-IR in the embryo contain RALDH-2 and synthesize retinoic acid. RALDH-2-IR indicates dynamic and discrete patterns of retinoic acid synthesis in the embryo, particularly within the somitic mesoderm, lateral mesoderm, kidney, heart, and spinal motor neurons. Prior to somitogenesis, RALDH-2-IR is present in the paraxial mesoderm with a rostral boundary at the level of the presumptive first somite; as the somites form, they exhibit strong RALDH-2-IR. Cervical presomitic mesoderm exhibits RALDH-2-IR but thoracic presomitic mesoderm does not. Neural crest cells do not express detectable levels of RALDH-2, but migrating crest cells are associated with RALDH-2 expressing mesoderm. The developing limb mesoderm expresses little RALDH-2-IR; however, RALDH-2-IR is strongly expressed in tissues adjacent to the limb. The most lateral, earliest-projecting motor neurons at all levels of the spinal cord exhibit RALDH-2-IR. Subsequently, many additional motor neurons in the brachial and lumbar cord regions express RALDH-2-IR. Motor neuronal expression of RALDH-2-IR is present in the growing axons as they extend to the periphery, indicating a potential role of retinoic acid in nerve influences on peripheral differentiation. With the exception of a transient expression in the facial/vestibulocochlear nucleus, cranial motor neurons do not express detectable levels of RALDH-2-IR.  (+info)

Chronic inflammatory demyelinating polyneuropathy with multiple hypertrophic nerves in intracranial, and intra- and extra-spinal segments. (2/248)

Hypertrophic nerves have occasionally been seen in chronic inflammatory demyelinating polyneuropathy (CIDP), but most are in the cauda equina. We report a case with CIDP in whom magnetic resonance imaging (MRI) with gadolinium diethylene triamine penta-acetic acid (Gd-DTPA) enhancement demonstrated hypertrophy of various peripheral nerves including multiple cranial nerves. Interestingly, none showed neurological signs corresponding to the lesions, except for clinical signs consistent with CIDP. MRI can be useful for the detection of silent, but abnormal nerve involvement in CIDP.  (+info)

Key roles of retinoic acid receptors alpha and beta in the patterning of the caudal hindbrain, pharyngeal arches and otocyst in the mouse. (3/248)

Mouse fetuses carrying targeted inactivations of both the RAR(&agr;) and the RARbeta genes display a variety of malformations in structures known to be partially derived from the mesenchymal neural crest originating from post-otic rhombomeres (e.g. thymus and great cephalic arteries) (Ghyselinck, N., Dupe, V., Dierich, A., Messaddeq, N., Garnier, J.M., Rochette-Egly, C., Chambon, P. and Mark M. (1997). Int. J. Dev. Biol. 41, 425-447). In a search for neural crest defects, we have analysed the rhombomeres, cranial nerves and pharyngeal arches of these double null mutants at early embryonic stages. The mutant post-otic cranial nerves are disorganized, indicating that RARs are involved in the patterning of structures derived from neurogenic neural crest, even though the lack of RARalpha and RARbeta has no detectable effect on the number and migration path of neural crest cells. Interestingly, the double null mutation impairs early developmental processes known to be independent of the neural crest e.g., the initial formation of the 3rd and 4th branchial pouches and of the 3rd, 4th and 6th arch arteries. The double mutation also results in an enlargement of rhombomere 5, which is likely to be responsible for the induction of supernumerary otic vesicles, in a disappearance of the rhombomere 5/6 boundary, and in profound alterations of rhombomere identities. In the mutant hindbrain, the expression domain of kreisler is twice its normal size and the caudal stripe of Krox-20 extends into the presumptive rhombomeres 6 and 7 region. In this region, Hoxb-1 is ectopically expressed, Hoxb-3 is ectopically up-regulated and Hoxd-4 expression is abolished. These data, which indicate that retinoic acid signaling through RARalpha and/or RARbeta is essential for the specification of rhombomere identities and for the control of caudal hindbrain segmentation by restricting the expression domains of kreisler and of Krox-20, also strongly suggest that this signaling plays a crucial role in the posteriorization of the hindbrain neurectoderm.  (+info)

Hepatocyte growth factor/scatter factor is a neurotrophic survival factor for lumbar but not for other somatic motoneurons in the chick embryo. (4/248)

Hepatocyte growth factor/scatter factor (HGF/SF) is expressed in the developing limb muscles of the chick embryo during the period of spinal motoneuron (MN) programmed cell death, and its receptor c-met is expressed in lumbar MNs during this same period. Although cultured motoneurons from brachial, thoracic, and lumbar segments are all rescued from cell death by chick embryo muscle extract (CMX) as well as by other specific trophic agents, HGF/SF only promotes the survival of lumbar MNs. Similarly, treatment of embryos in ovo with exogenous HGF/SF rescues lumbar but not other somatic MNs from cell death. Blocking antibodies to HGF/SF (anti-HGF) reduce the effects of CMX on MN survival in vitro and decrease the number of lumbar MNs in vivo. The expression of c-met on MNs in vivo is regulated by a limb-derived trophic signal distinct from HGF/SF. HGF/SF is a potent, select, and physiologically relevant survival factor for a subpopulation of developing spinal MNs in the lumbar segments of the chick embryo.  (+info)

Neuropilin-2 regulates the development of selective cranial and sensory nerves and hippocampal mossy fiber projections. (5/248)

Neuropilin-1 and neuropilin-2 bind differentially to different class 3 semaphorins and are thought to provide the ligand-binding moieties in receptor complexes mediating repulsive responses to these semaphorins. Here, we have studied the function of neuropilin-2 through analysis of a neuropilin-2 mutant mouse, which is viable and fertile. Repulsive responses of sympathetic and hippocampal neurons to Sema3F but not to Sema3A are abolished in the mutant. Marked defects are observed in the development of several cranial nerves, in the initial central projections of spinal sensory axons, and in the anterior commissure, habenulo-interpeduncular tract, and the projections of hippocampal mossyfiber axons in the infrapyramidal bundle. Our results show that neuropilin-2 is an essential component of the Sema3F receptor and identify key roles for neuropilin-2 in axon guidance in the PNS and CNS.  (+info)

Vitamin A deficiency results in the dose-dependent acquisition of anterior character and shortening of the caudal hindbrain of the rat embryo. (6/248)

The developing nervous system is particularly vulnerable to vitamin A deficiency. Retinoid has been proposed to be a posteriorizing factor during hindbrain development, although direct evidence in the mammalian embryo is lacking. In the present study, pregnant vitamin A-deficient (VAD) rats were fed purified diets containing varying levels of all-trans-retinoic acid (atRA; 0, 0.5, 1.5, 6, 12, 25, 50, 125, or 250 microg/g diet) or were supplemented with retinol. Hindbrain development was studied from embryonic day 10 to 12.5 ( approximately 6 to 40 somites). Normal morphogenesis was observed in all embryos from groups fed 250 microg atRA/g diet or retinol. The most caudal region of the hindbrain was the most sensitive to retinoid insufficiency, as evidenced by a loss of the hypoglossal nerve (cranial nerve XII) in embryos from the 125 microg atRA/g diet group. Further reduction of atRA to 50 microg/g diet led to the loss of cranial nerves IX, X, XI, and XII and associated sensory ganglia IX and X in all embryos as well as the loss of hindbrain segmentation caudal to the rhombomere (r) 3/4 border in a subset of embryos. Dysmorphic orthotopic otic vesicles or immature otic-like vesicles in both orthotopic and caudally ectopic locations were also observed. As the level of atRA was reduced, a loss of caudal hindbrain segmentation was observed in all embryos and the incidence of otic vesicle abnormalities increased. Perturbations in hindbrain segmentation, cranial nerve formation, and otic vesicle development were associated with abnormal patterning of the posterior hindbrain. Embryos from VAD dams fed between 0.5 and 50 microg atRA/g diet exhibited Hoxb-1 protein expression along the entire neural tube caudal to the r3/r4 border at a time when it should be restricted to r4. Krox-20 protein expression was expanded in r3 but absent or reduced in presumptive r5. Hoxd-4 mRNA expression was absent in the posterior hindbrain, and the rostral limit of Hoxb-5 protein expression in the neural tube was anteriorized, suggesting that the most posterior hindbrain region (r7/r8) had been deleted and/or improperly patterned. Thus, when limiting amounts of atRA are provided to VAD dams, the caudal portion of the hindbrain is shortened and possesses r4/r5-like characteristics, with this region finally exhibiting r4-like gene expression when retinoid is restricted even more severely. Thus, regions of the anterior hindbrain (i.e., r3 and r4) appear to be greatly expanded, whereas the posterior hindbrain (r5-r8) is reduced or absent. This work shows that retinoid plays a critical role in patterning, segmentation, and neurogenesis of the caudal hindbrain and serves as an essential posteriorizing signal for this region of the central nervous system in the mammal.  (+info)

Development of the cavernous sinus in the fetal period: a morphological study. (7/248)

The development and morphological structure of the lateral sellar compartment (LSC), an interdural space containing the cavernous sinus (CS), cranial nerves, and internal carotid artery (ICA), was investigated by histological examination of sections of the LSC and cerebral venograms from human fetal specimens. Twenty-eight LSC coronal sections were obtained from 14 fetuses of 13-32 weeks' gestation. Venograms of 11 other fetuses of 13-32 weeks' gestation were studied to observe changes in venous drainage. The CS appeared as a collection of small venous canals with an endothelial layer. These venous canals gradually became much larger through expansion and unification. The CS and basilar venous plexus were demonstrated as a faint cluster of small vessels on venograms obtained after 13 weeks' gestation. The dura mater increased in thickness and collagen fiber networks developed around all the components in the LSC after 23 weeks' gestation. The LSC lateral wall could not be histologically differentiated as separate multiple layers. Branching and joining of the cranial nerve fascicles were completed with the envelopment of collagen fibers after 23 weeks' gestation. The ICA at 13-15 weeks' gestation ran straight within the LSC, becoming tortuous before birth. CS formation occurs through the development of venous canals without smooth muscle layers, followed by web-formation by collagen fibers in the mesenchymal interstices. LSC formation, including the dense dura mater and an internal structure like that seen in the adult, is largely completed before birth.  (+info)

Facial palsy in cerebral venous thrombosis : transcranial stimulation and pathophysiological considerations. (8/248)

BACKGROUND: Cranial nerve palsy in cerebral sinovenous thrombosis (CVT) is rare, its pathophysiology remains unclear, and data from electrophysiological examinations in such patients are missing. CASE DESCRIPTION: We report the case of a 17-year-old woman with familial protein S deficiency who was admitted with extensive multiple CVT. Two weeks after onset of symptoms, she developed isolated right peripheral facial palsy, and MR venography showed segmental occlusion of the ipsilateral transverse sinus. Complete recovery of facial palsy occurred concomitant with recanalization of the transverse sinus. Facial neurography, including transcranial magnetic stimulation of the facial nerve and related motor cortex, ruled out a coincidental idiopathic palsy and revealed conduction block proximal to the facial canal. CONCLUSIONS: Facial palsy in our patient was caused by transient neurapraxia in the intracranial segment of the nerve. We suggest that elevated venous transmural pressure in the nerve's satellite vein, which belongs to the affected drainage territory of the transverse sinus, might have caused venous blood-brain barrier dysfunction in the intrinsic vascular system of the nerve, with leakage of fluids and ions into the endoneurial space and thus an increase in interstitial resistance.  (+info)

Some common examples of cranial nerve diseases include:

1. Bell's palsy: A condition that affects the facial nerve, causing weakness or paralysis of one side of the face.
2. Multiple sclerosis: An autoimmune disease that damages the protective covering of nerve fibers, leading to communication problems between the brain and the rest of the body.
3. Trigeminal neuralgia: A condition that affects the trigeminal nerve, causing facial pain and numbness.
4. Meningitis: An inflammation of the meninges, the protective covering of the brain and spinal cord, which can damage the cranial nerves.
5. Acoustic neuroma: A type of non-cancerous tumor that grows on the nerve that connects the inner ear to the brain.
6. Cranial polyneuropathy: A condition where multiple cranial nerves are damaged, leading to a range of symptoms including muscle weakness, numbness, and pain.
7. Tumors: Both benign and malignant tumors can affect the cranial nerves, causing a variety of symptoms depending on their location and size.
8. Trauma: Head injuries or trauma can damage the cranial nerves, leading to a range of symptoms.
9. Infections: Bacterial or viral infections such as meningitis or encephalitis can damage the cranial nerves, leading to a range of symptoms.
10. Genetic disorders: Certain genetic disorders such as Charcot-Marie-Tooth disease can affect the cranial nerves, leading to a range of symptoms.

It's important to note that this is not an exhaustive list and there may be other causes of cranial nerve damage. If you are experiencing any symptoms that you think may be related to cranial nerve damage, it's important to seek medical attention as soon as possible for proper diagnosis and treatment.

Types of Cranial Nerve Injuries:

1. Traumatic brain injury (TBI): TBI can cause damage to the cranial nerves, leading to a range of symptoms such as double vision, facial weakness or paralysis, difficulty with swallowing, and cognitive impairment.
2. Stroke: A stroke can cause damage to the cranial nerves, leading to symptoms such as a drooping eyelid, facial weakness or paralysis, and difficulty with swallowing.
3. Brain tumors: Tumors in the brain can compress or damage the cranial nerves, causing a range of symptoms such as double vision, facial weakness or paralysis, and cognitive impairment.
4. Cerebral vasospasm: This is a condition where the blood vessels in the brain constrict, reducing blood flow and oxygen supply to the brain, which can cause damage to the cranial nerves.
5. Infections such as meningitis or encephalitis: These infections can cause inflammation of the membranes surrounding the brain and spinal cord, leading to damage to the cranial nerves.
6. Neurodegenerative diseases such as Parkinson's disease, multiple sclerosis, and amyotrophic lateral sclerosis (ALS): These conditions can cause progressive damage to the cranial nerves leading to a range of symptoms such as tremors, weakness, and difficulty with movement and balance.

Symptoms of Cranial Nerve Injuries:

1. Double vision or loss of vision
2. Facial weakness or paralysis
3. Difficulty with swallowing
4. Slurred speech
5. Weakness or paralysis of the limbs on one side of the body
6. Difficulty with balance and coordination
7. Numbness or tingling in the face, arms, or legs
8. Seizures
9. Vision problems such as blurred vision, loss of peripheral vision, or loss of color vision
10. Cognitive impairment such as difficulty with concentration, memory loss, or difficulty with problem-solving.

Diagnosis of Cranial Nerve Injuries:

1. Physical examination and medical history: A doctor will perform a physical examination to check for signs of cranial nerve damage such as weakness or paralysis of the facial muscles, difficulty with swallowing, or abnormal reflexes.
2. Imaging tests such as CT or MRI scans: These tests can help doctors identify any structural problems in the brain or spinal cord that may be causing cranial nerve damage.
3. Electromyography (EMG) and nerve conduction studies (NCS): These tests can help doctors determine the extent of nerve damage by measuring the electrical activity of muscles and nerves.
4. Lumbar puncture: This test involves inserting a needle into the spinal canal to collect cerebrospinal fluid for laboratory testing.
5. Blood tests: These can help doctors rule out other conditions that may be causing symptoms such as infections or autoimmune disorders.

Treatment of Cranial Nerve Injuries:

1. Conservative management: Mild cases of cranial nerve injuries may not require surgical intervention and can be treated with conservative measures such as physical therapy, pain management, and monitoring.
2. Surgery: In more severe cases, surgery may be necessary to relieve compression on the nerves or repair any structural damage.
3. Rehabilitation: After surgery or conservative treatment, rehabilitation is crucial to regain lost function and prevent further complications. This may include physical therapy, occupational therapy, and speech therapy.

Prognosis of Cranial Nerve Injuries:

The prognosis for cranial nerve injuries depends on the severity and location of the injury, as well as the promptness and effectiveness of treatment. In general, the sooner treatment is received, the better the outcome. Some people may experience a full recovery, while others may have persistent symptoms or long-term deficits.

Complications of Cranial Nerve Injuries:

1. Permanent nerve damage: In some cases, cranial nerve injuries can result in permanent nerve damage, leading to chronic symptoms such as weakness, numbness, or paralysis.
2. Seizures: Cranial nerve injuries can increase the risk of seizures, particularly if they involve the seizure-regulating nerves.
3. Infection: Any injury that penetrates the skull can increase the risk of infection, which can be life-threatening if left untreated.
4. Hydrocephalus: This is a condition in which cerebrospinal fluid accumulates in the brain, leading to increased intracranial pressure and potentially life-threatening complications.
5. Cerebral edema: This is swelling of the brain tissue due to injury or inflammation, which can lead to increased intracranial pressure and potentially life-threatening complications.
6. Brain herniation: This is a condition in which the brain is pushed out of its normal position in the skull, leading to potentially life-threatening complications.
7. Vision loss: Cranial nerve injuries can cause vision loss or blindness, particularly if they involve the optic nerves.
8. Facial paralysis: Cranial nerve injuries can cause facial paralysis or weakness, which can be temporary or permanent.
9. Hearing loss: Cranial nerve injuries can cause hearing loss or deafness, particularly if they involve the auditory nerves.
10. Cognitive and behavioral changes: Depending on the location and severity of the injury, cranial nerve injuries can lead to cognitive and behavioral changes, such as difficulty with concentration, memory problems, or personality changes.

In summary, cranial nerve injuries can have a significant impact on an individual's quality of life, and it is important to seek medical attention immediately if symptoms persist or worsen over time.

The symptoms of cranial nerve neoplasms depend on the location and size of the tumor, but may include:

* Headaches
* Pain in the face or head
* Numbness or weakness in the arms or legs
* Difficulty with vision, hearing, or balance
* Double vision
* Nausea and vomiting

Cranial nerve neoplasms can be diagnosed through a variety of tests, including:

* Imaging studies such as MRI or CT scans
* Biopsy, where a sample of tissue is removed for examination under a microscope
* Neurological examination to assess vision, hearing, balance, and other functions.

Treatment options for cranial nerve neoplasms depend on the location, size, and type of tumor, as well as the patient's overall health. Treatment may include:

* Surgery to remove the tumor
* Radiation therapy to kill any remaining cancer cells
* Chemotherapy to kill cancer cells
* Targeted therapy to attack specific molecules on the surface of cancer cells
* Observation, with regular monitoring and check-ups to see if the tumor is growing or changing.

It's important to note that cranial nerve neoplasms are relatively rare, and the prognosis and treatment options can vary depending on the specific type of tumor and the patient's overall health. A healthcare professional should be consulted for an accurate diagnosis and appropriate treatment plan.

Some common abducens nerve diseases include:

1. Abducens paresis or palsy: This is a weakness or paralysis of the abducens nerve that can cause difficulty moving the eyeball outward or away from the nose.
2. Brown syndrome: This is a condition where the nerve is compressed or damaged, leading to difficulty moving the eye laterally.
3. Congenital abducens palsy: This is a condition present at birth that affects the development of the abducens nerve and can result in limited or absent movement of one or both eyes.
4. Trauma to the abducens nerve: This can occur due to head injuries, facial trauma, or other forms of injury that damage the nerve.
5. Tumors or cysts: Growths in the orbit or near the abducens nerve can compress or damage the nerve and cause abducens nerve diseases.
6. Inflammatory conditions: Conditions such as Graves' disease, multiple sclerosis, or sarcoidosis can inflame the nerve and cause abducens nerve diseases.
7. Stroke or cerebral vasculature disorders: These conditions can damage the nerve due to reduced blood flow or bleeding in the brain.

Symptoms of abducens nerve diseases may include double vision, difficulty moving one or both eyes, and difficulty focusing. Diagnosis is typically made through a combination of physical examination, imaging studies such as MRI or CT scans, and electrophysiological tests such as electromyography. Treatment options vary depending on the underlying cause of the disease and may include glasses or contact lenses for double vision, prism lenses to align the eyes, or surgery to correct any anatomical abnormalities. In some cases, medications such as steroids or immunosuppressants may be prescribed to reduce inflammation and promote healing.

Damage or dysfunction of the oculomotor nerve can result in a range of symptoms, including double vision (diplopia), drooping eyelids (ptosis), difficulty moving the eyes (ophthalmoplegia), and vision loss. The specific symptoms depend on the location and extent of the damage to the nerve.

Some common causes of oculomotor nerve diseases include:

1. Trauma or injury to the head or neck
2. Tumors or cysts in the brain or skull
3. Inflammatory conditions such as multiple sclerosis or sarcoidosis
4. Vasculitis or other blood vessel disorders
5. Certain medications, such as anticonvulsants or chemotherapy drugs
6. Nutritional deficiencies, such as vitamin B12 deficiency
7. Infections, such as meningitis or encephalitis
8. Genetic disorders, such as hereditary oculopharyngeal dystrophy
9. Ischemic or hemorrhagic strokes
10. Neurodegenerative diseases, such as Parkinson's disease or amyotrophic lateral sclerosis (ALS).

The diagnosis of oculomotor nerve diseases typically involves a comprehensive eye exam, neurological evaluation, and imaging studies such as MRI or CT scans. Treatment depends on the underlying cause and may include medications, surgery, or other interventions to address the underlying condition and relieve symptoms. In some cases, surgical intervention may be necessary to repair or replace damaged portions of the nerve.

The hypoglossal nerve is a cranial nerve that controls the movement of the tongue and is responsible for its protrusion, withdrawal, and lateral movement. Hypoglossal nerve injuries can occur due to various reasons such as trauma, surgery, or tumors. These injuries can result in symptoms such as tongue weakness or paralysis, difficulty speaking or swallowing, and loss of taste sensation on the tip of the tongue.

The severity of hypoglossal nerve injuries can vary from mild to severe, and the treatment options depend on the cause and extent of the injury. Mild cases may resolve on their own with time, while more severe cases may require surgical intervention or other treatments such as physical therapy or medications. In this article, we will discuss the causes, symptoms, diagnosis, and treatment options for hypoglossal nerve injuries in detail.

Causes of Hypoglossal Nerve Injuries:

Hypoglossal nerve injuries can occur due to various reasons such as:

Trauma: Traumatic injuries to the face or neck can cause damage to the hypoglossal nerve, resulting in tongue weakness or paralysis.

Surgery: Surgical procedures in the head and neck region can sometimes result in injury to the hypoglossal nerve.

Tumors: Tumors in the head and neck region can compress or injure the hypoglossal nerve, leading to tongue weakness or paralysis.

Infections: Infections such as meningitis or abscesses in the head and neck region can damage the hypoglossal nerve.

Neurodegenerative diseases: Certain neurodegenerative diseases such as amyotrophic lateral sclerosis (ALS) can affect the hypoglossal nerve, leading to tongue weakness or paralysis.

Symptoms of Hypoglossal Nerve Injuries:

The symptoms of hypoglossal nerve injuries can vary depending on the severity and location of the injury. Common symptoms include:

Tongue weakness or paralysis: Weakness or paralysis of the tongue can make it difficult to speak, eat, or swallow.

Drooling: Inability to control salivation due to tongue weakness or paralysis can lead to drooling.

Difficulty articulating words: Slurred speech or difficulty articulating words due to tongue weakness or paralysis.

Facial weakness or paralysis: Weakness or paralysis of the facial muscles can cause drooping or weakness in the face.

Difficulty swallowing: Difficulty swallowing due to tongue weakness or paralysis can lead to dysphagia.

Causes of Hypoglossal Nerve Injuries:

Hypoglossal nerve injuries can occur due to various reasons, including:

Trauma: Traumatic injuries to the face or neck can cause damage to the hypoglossal nerve, resulting in tongue weakness or paralysis.

Surgery: Surgical procedures in the head and neck region can sometimes cause nerve damage, leading to hypoglossal nerve injuries.

Neurological conditions: Certain neurological conditions such as stroke, multiple sclerosis, or tumors can cause hypoglossal nerve injuries.

Viral infections: Viral infections such as HIV or Lyme disease can cause inflammation of the nerves, including the hypoglossal nerve.

Treatment options for Hypoglossal Nerve Injuries:

Treatment options for hypoglossal nerve injuries depend on the underlying cause and severity of the injury. Some possible treatment options include:

Physical therapy: Physical therapy exercises can help improve tongue strength and mobility.

Medications: Medications such as antiviral drugs or steroids may be prescribed to manage symptoms.

Surgery: In some cases, surgery may be necessary to relieve compression or repair damaged nerve tissue.

Speech therapy: Speech therapy can help improve communication skills and address swallowing difficulties.

It's important to seek medical attention if you experience any symptoms of hypoglossal nerve injuries, as prompt treatment can help prevent long-term complications and improve outcomes.

The symptoms of Mobius syndrome are caused by damage to the sixth and seventh cranial nerves, which control facial movements and swallowing. The disorder is usually inherited as an autosomal dominant trait, meaning that a single copy of the mutated gene is enough to cause the condition. However, some cases may occur spontaneously due to a genetic mutation or other factors.

There is no cure for Mobius syndrome, but treatment can help manage the symptoms and improve quality of life. Physical therapy, occupational therapy, and speech therapy may be recommended to help improve muscle strength and coordination, as well as communication skills. In some cases, surgery may be necessary to correct physical deformities or relieve pressure on the brain and spinal cord.

The prognosis for Mobius syndrome varies depending on the severity of the condition and the presence of any other underlying health issues. Some individuals with Mobius syndrome may have a relatively mild form of the disorder, while others may experience more severe symptoms and functional limitations. With appropriate treatment and support, many people with Mobius syndrome are able to lead fulfilling lives and achieve their goals.

There are several types of nerve compression syndromes, including:

1. Carpal tunnel syndrome: Compression of the median nerve in the wrist, commonly caused by repetitive motion or injury.
2. Tarsal tunnel syndrome: Compression of the posterior tibial nerve in the ankle, similar to carpal tunnel syndrome but affecting the lower leg.
3. Cubital tunnel syndrome: Compression of the ulnar nerve at the elbow, often caused by repetitive leaning or bending.
4. Thoracic outlet syndrome: Compression of the nerves and blood vessels that pass through the thoracic outlet (the space between the neck and shoulder), often caused by poor posture or injury.
5. Peripheral neuropathy: A broader term for damage to the peripheral nerves, often caused by diabetes, vitamin deficiencies, or other systemic conditions.
6. Meralgia paresthetica: Compression of the lateral femoral cutaneous nerve in the thigh, commonly caused by direct trauma or compression from a tight waistband or clothing.
7. Morton's neuroma: Compression of the plantar digital nerves between the toes, often caused by poorly fitting shoes or repetitive stress on the feet.
8. Neuralgia: A general term for pain or numbness caused by damage or irritation to a nerve, often associated with chronic conditions such as shingles or postherpetic neuralgia.
9. Trigeminal neuralgia: A condition characterized by recurring episodes of sudden, extreme pain in the face, often caused by compression or irritation of the trigeminal nerve.
10. Neuropathic pain: Pain that occurs as a result of damage or dysfunction of the nervous system, often accompanied by other symptoms such as numbness, tingling, or weakness.

There are several types of ophthalmoplegia, including:

1. External ophthalmoplegia: This type affects the muscles that control lateral and vertical movements of the eyes.
2. Internal ophthalmoplegia: This type affects the muscles that control rotational movements of the eyes.
3. Superior oblique paresis: This type affects the superior oblique muscle, which controls downward and outward movements of the eye.
4. Inferior oblique paresis: This type affects the inferior oblique muscle, which controls upward and outward movements of the eye.

Symptoms of ophthalmoplegia may include difficulty moving the eyes, double vision, droopy eyelids, and blurred vision. Treatment options depend on the underlying cause of the condition and may include physical therapy, prism lenses, or surgery.

The main symptoms of facial paralysis are:

1. Weakness or numbness in the facial muscles
2. Drooping or sagging of one side of the face
3. Twitching or spasms in the facial muscles
4. Difficulty smiling, frowning, or expressing emotions
5. Difficulty closing the eye on the affected side
6. Dry mouth or difficulty swallowing
7. Pain or discomfort in the face or head.

The diagnosis of facial paralysis is based on a combination of clinical examination, imaging studies such as MRI or CT scans, and other tests to determine the underlying cause. Treatment options for facial paralysis depend on the underlying cause and may include medications, surgery, physical therapy, and other interventions to address any associated symptoms.

There are several types of facial paralysis, including:

1. Bell's palsy: A condition that causes weakness or paralysis of the muscles on one side of the face, usually due to nerve damage.
2. Facial spasm: A condition characterized by involuntary twitching or contractions of the facial muscles.
3. Hemifacial spasm: A condition that causes weakness or paralysis of half of the face due to nerve compression.
4. Trauma-related facial paralysis: Caused by injury or trauma to the face or head.
5. Tumor-related facial paralysis: Caused by a tumor that compresses or damages the nerves responsible for facial movement.
6. Stroke-related facial paralysis: Caused by a stroke that affects the nerves responsible for facial movement.
7. Neurodegenerative diseases such as Parkinson's disease, multiple sclerosis, and amyotrophic lateral sclerosis (ALS).
8. Infection-related facial paralysis: Caused by infections such as Lyme disease, meningitis, or encephalitis.
9. Post-viral facial paralysis: Caused by a viral infection that affects the nerves responsible for facial movement.

Treatment for facial paralysis depend on the underlying cause and may include medications, surgery, physical therapy, and other interventions to address any associated symptoms.

The trigeminal nerve is a cranial nerve that carries sensation from the face and head to the brain. Trigeminal nerve diseases are conditions that affect this nerve, leading to a range of symptoms such as pain, numbness, weakness, and difficulty with facial movements.

Types of Trigeminal Nerve Diseases:

1. Trigeminal Neuralgia: This is a chronic pain disorder that affects the trigeminal nerve, causing episodes of sudden, intense pain in the face, particularly around the eye and mouth.
2. Multiple Sclerosis (MS): MS is an autoimmune disease that can damage the trigeminal nerve, leading to pain, numbness, and weakness in the face.
3. Trigeminal Neuropathy: This is a condition where the trigeminal nerve is damaged due to injury, inflammation, or infection, leading to pain, numbness, and tingling in the face.
4. Bell's Palsy: This is a condition that affects the facial nerve, leading to weakness or paralysis of the muscles on one side of the face.
5. Herpes Zoster Oticus: This is a viral infection that affects the nerves in the ear and face, causing pain, numbness, and tingling in the face.

Symptoms of Trigeminal Nerve Diseases:

1. Pain: The most common symptom of trigeminal nerve diseases is pain, which can range from mild to severe and can be described as aching, burning, or electric-like.
2. Numbness or tingling: Patients may experience numbness or tingling sensations in the face, particularly around the eye and mouth.
3. Weakness: Some patients may experience weakness or paralysis of the muscles in the face, which can affect their ability to smile, talk, or eat.
4. Difficulty with facial movements: Trigeminal nerve diseases can cause difficulty with facial movements such as closing the eyes, smiling, or whistling.
5. Drooping eyelid or eyebrow: Some patients may experience drooping of the eyelid or eyebrow, which can be a sign of a more severe condition.
6. Eye problems: Trigeminal nerve diseases can cause eye problems such as double vision, blurred vision, or loss of vision in one eye.
7. Headaches: Patients may experience headaches or migraines due to the pressure or inflammation on the nerve.
8. Fatigue: Trigeminal nerve diseases can cause fatigue and exhaustion, particularly if the patient is experiencing persistent pain or discomfort.

Diagnosis of Trigeminal Nerve Diseases:

1. Medical history and physical examination: A thorough medical history and physical examination are essential to diagnose trigeminal nerve diseases.
2. Imaging studies: Imaging studies such as MRI or CT scans may be ordered to rule out other conditions and visualize the nerve.
3. Nerve conduction studies: Nerve conduction studies can help identify the specific location and extent of the nerve damage.
4. Blood tests: Blood tests may be ordered to check for signs of inflammation or infection.
5. Biopsy: A biopsy may be performed to obtain a tissue sample for further examination.

Treatment of Trigeminal Nerve Diseases:

1. Pain management: Pain management is the primary goal of treatment, and it can be achieved through medications, injections, or nerve blocks.
2. Anticonvulsants: Anticonvulsants may be prescribed to manage pain and prevent seizures.
3. Anti-inflammatory medications: Anti-inflammatory medications may be used to reduce inflammation and swelling.
4. Muscle relaxants: Muscle relaxants may be prescribed to relieve muscle spasms and tension.
5. Physical therapy: Physical therapy can help improve range of motion, strength, and function.
6. Surgery: In some cases, surgery may be necessary to relieve compression or damage to the nerve.

Prevention of Trigeminal Nerve Diseases:

1. Early diagnosis and treatment: Early diagnosis and treatment can help prevent progression of the disease and reduce symptoms.
2. Avoiding triggers: Avoiding triggers such as allergens, infections, or trauma can help prevent the onset of trigeminal nerve diseases.
3. Good oral hygiene: Maintaining good oral hygiene can help prevent infections that can lead to trigeminal nerve damage.
4. Avoiding repetitive motions: Avoiding repetitive motions such as frequent clenching or grinding of the teeth can help prevent nerve damage.
5. Proper body mechanics: Maintaining proper body mechanics and posture can help reduce strain on the nerve.
6. Regular check-ups: Regular check-ups with a healthcare professional can help detect any underlying conditions and prevent complications.

Some examples of vestibulocochlear nerve diseases include:

1. Meniere's disease: A disorder of the inner ear that causes vertigo, tinnitus, hearing loss, and a feeling of fullness in the affected ear.
2. Acoustic neuroma: A benign tumor that grows on the vestibular nerve and can cause hearing loss, tinnitus, and balance difficulties.
3. Otosclerosis: A condition in which there is abnormal bone growth in the middle ear that can cause hearing loss and tinnitus.
4. Presbycusis: Age-related hearing loss that affects the inner ear and causes gradual hearing loss over time.
5. Sudden sensorineural hearing loss: A condition where an individual experiences sudden and significant hearing loss in one or both ears with no known cause.
6. Meningitis: Inflammation of the membranes that cover the brain and spinal cord, which can affect the vestibulocochlear nerve and cause hearing loss and balance difficulties.
7. Certain medications: Certain antibiotics, chemotherapy drugs, and aspirin at high doses can damage the inner ear and cause temporary or permanent hearing loss.
8. Trauma to the head or ear: A head injury or a sudden blow to the ear can cause damage to the vestibulocochlear nerve and result in hearing loss or balance difficulties.

These are some of the common examples of vestibulocochlear nerve diseases, but there are other rarer conditions that can also affect the vestibulocochlear nerve. A comprehensive evaluation by an otolaryngologist (ENT specialist) and a hearing specialist is necessary for proper diagnosis and treatment.

The symptoms of TN can vary in severity and frequency, and may include:

* Pain on one side of the face
* Episodes of sudden, intense pain that can be triggered by light touch or contact with the face
* Pain that is described as stabbing, shooting, or like an electric shock
* Spontaneous pain episodes without any apparent cause
* Pain that is worse with light sensation, such as from wind, cold, or touch
* Pain that is better with pressing or rubbing the affected area

The exact cause of TN is not known, but it is believed to be related to compression or irritation of the trigeminal nerve. The condition can be caused by a variety of factors, including:

* A blood vessel pressing on the nerve
* A tumor or cyst in the brain or face
* Multiple sclerosis or other conditions that damage the nerve
* Injury to the nerve
* Genetic mutations that affect the nerve

There is no cure for TN, but various treatments can help manage the symptoms. These may include:

* Medications such as anticonvulsants or pain relievers
* Nerve blocks or injections to reduce inflammation and relieve pain
* Surgery to decompress the nerve or remove a tumor or cyst
* Lifestyle modifications, such as avoiding triggers and using gentle, soothing touch

It is important for individuals with TN to work closely with their healthcare provider to find the most effective treatment plan for their specific needs. With proper management, many people with TN are able to experience significant relief from their symptoms and improve their quality of life.

1. Complete paralysis: When there is no movement or sensation in a particular area of the body.
2. Incomplete paralysis: When there is some movement or sensation in a particular area of the body.
3. Localized paralysis: When paralysis affects only a specific part of the body, such as a limb or a facial muscle.
4. Generalized paralysis: When paralysis affects multiple parts of the body.
5. Flaccid paralysis: When there is a loss of muscle tone and the affected limbs feel floppy.
6. Spastic paralysis: When there is an increase in muscle tone and the affected limbs feel stiff and rigid.
7. Paralysis due to nerve damage: This can be caused by injuries, diseases such as multiple sclerosis, or birth defects such as spina bifida.
8. Paralysis due to muscle damage: This can be caused by injuries, such as muscular dystrophy, or diseases such as muscular sarcopenia.
9. Paralysis due to brain damage: This can be caused by head injuries, stroke, or other conditions that affect the brain such as cerebral palsy.
10. Paralysis due to spinal cord injury: This can be caused by trauma, such as a car accident, or diseases such as polio.

Paralysis can have a significant impact on an individual's quality of life, affecting their ability to perform daily activities, work, and participate in social and recreational activities. Treatment options for paralysis depend on the underlying cause and may include physical therapy, medications, surgery, or assistive technologies such as wheelchairs or prosthetic devices.

Some examples of Facial Nerve Diseases include:

* Bell's Palsy: A condition that causes weakness or paralysis of the facial muscles on one side of the face, often resulting in drooping or twitching of the eyelid and facial muscles.
* Facial Spasm: A condition characterized by involuntary contractions of the facial muscles, which can cause twitching or spasms.
* Progressive Bulbar Palsy (PBP): A rare disorder that affects the brain and spinal cord, leading to weakness and wasting of the muscles in the face, tongue, and throat.
* Parry-Romberg Syndrome: A rare condition characterized by progressive atrophy of the facial muscles on one side of the face, leading to a characteristic "smile" or "grimace."
* Moebius Syndrome: A rare neurological disorder that affects the nerves responsible for controlling eye movements and facial expressions.
* Trauma to the Facial Nerve: Damage to the facial nerve can result in weakness or paralysis of the facial muscles, depending on the severity of the injury.

These are just a few examples of Facial Nerve Diseases, and there are many other conditions that can affect the facial nerve and cause similar symptoms. A comprehensive diagnosis and evaluation by a healthcare professional is necessary to determine the specific underlying condition and develop an appropriate treatment plan.

Types of Skull Base Neoplasms:

1. Meningioma: A benign tumor that arises from the meninges, the protective membranes covering the brain and spinal cord.
2. Acoustic neuroma (vestibular schwannoma): A benign tumor that grows on the nerve that connects the inner ear to the brain.
3. Pineal parenchymal tumors: Tumors that occur in the pineal gland, a small endocrine gland located in the brain.
4. Craniopharyngiomas: Benign tumors that arise from the cells of the pituitary gland and the hypothalamus.
5. Chordomas: Malignant tumors that arise from the cells of the notochord, a structure that gives rise to the spinal cord.
6. Chondrosarcomas: Malignant tumors that arise from cartilage cells.
7. Osteosarcomas: Malignant tumors that arise from bone cells.
8. Melanotic neuroectodermal tumors: Rare tumors that are usually benign but can sometimes be malignant.

Causes and Symptoms of Skull Base Neoplasms:

The exact cause of skull base neoplasms is not always known, but they can be associated with genetic mutations or exposure to certain environmental factors. Some of the symptoms of skull base neoplasms include:

* Headaches
* Vision problems
* Hearing loss
* Balance and coordination difficulties
* Seizures
* Weakness or numbness in the face or limbs
* Endocrine dysfunction (in case of pituitary tumors)

Diagnosis of Skull Base Neoplasms:

The diagnosis of skull base neoplasms usually involves a combination of imaging studies such as CT or MRI scans, and tissue sampling through biopsy or surgery. The specific diagnostic tests will depend on the location and symptoms of the tumor.

Treatment of Skull Base Neoplasms:

The treatment of skull base neoplasms depends on the type, size, location, and aggressiveness of the tumor, as well as the patient's overall health. Some of the treatment options for skull base neoplasms include:

* Surgery: The primary treatment for most skull base neoplasms is surgical resection. The goal of surgery is to remove as much of the tumor as possible while preserving as much normal tissue as possible.
* Radiation therapy: Radiation therapy may be used before or after surgery to shrink the tumor and kill any remaining cancer cells.
* Chemotherapy: Chemotherapy may be used in combination with radiation therapy to treat skull base neoplasms that are aggressive or have spread to other parts of the body.
* Endoscopic surgery: Endoscopic surgery is a minimally invasive procedure that uses a thin, lighted tube with a camera on the end (endoscope) to remove the tumor through the nasal cavity or sinuses.
* Stereotactic radiosurgery: Stereotactic radiosurgery is a non-invasive procedure that uses highly focused radiation beams to destroy the tumor. It is typically used for small, well-defined tumors that are located in sensitive areas of the skull base.

Prognosis for Skull Base Neoplasms:

The prognosis for skull base neoplasms depends on the type and location of the tumor, as well as the patient's overall health. In general, the prognosis for patients with skull base neoplasms is good if the tumor is small, located in a accessible area, and has not spread to other parts of the body. However, the prognosis may be poorer for patients with larger or more aggressive tumors, or those that have spread to other parts of the body.

It's important to note that each patient is unique and the prognosis can vary depending on individual circumstances. It is best to consult a medical professional for specific information about the prognosis for your condition.

There are several types of facial nerve injuries, including:

1. Bell's palsy: This is a condition that affects the facial nerve and causes weakness or paralysis of the muscles on one side of the face. It is often temporary and resolves on its own within a few weeks.
2. Facial paralysis: This is a condition in which the facial nerve is damaged, leading to weakness or paralysis of the muscles of facial expression. It can be caused by trauma, tumors, or viral infections.
3. Ramsay Hunt syndrome: This is a rare condition that occurs when the facial nerve is affected by a virus, leading to symptoms such as facial paralysis and pain in the ear.
4. Traumatic facial nerve injury: This can occur as a result of trauma to the head or face, such as a car accident or a fall.
5. Tumor-related facial nerve injury: In some cases, tumors can grow on the facial nerve and cause damage.
6. Ischemic facial nerve injury: This occurs when there is a reduction in blood flow to the facial nerve, leading to damage to the nerve fibers.
7. Neurofibromatosis type 2: This is a rare genetic disorder that can cause tumors to grow on the facial nerve, leading to damage and weakness of the facial muscles.

Treatment for facial nerve injuries depends on the underlying cause and severity of the injury. In some cases, physical therapy may be recommended to help regain strength and control of the facial muscles. Surgery may also be necessary in some cases to repair damaged nerve fibers or remove tumors.

The trochlear nerve is a small nerve that originates in the brain and passes through the base of the skull, down through the orbit (eye socket), and into the eye. It provides sensation to the superior oblique muscle, which is responsible for rotating the eye downward and outward. Injuries to the trochlear nerve can cause a variety of symptoms, including double vision, drooping of the eyelid, and difficulty moving the eyes.

The most common causes of trochlear nerve injuries include:

1. Trauma to the head or eye
2. Inflammation of the orbit (orbital inflammation)
3. Tumors in the orbit or brain
4. Cranial or orbital fractures
5. Infections such as meningitis or encephalitis
6. Stroke or cerebral vasculature disorders
7. Neurodegenerative diseases such as Parkinson's disease, multiple sclerosis, or myasthenia gravis.

Symptoms of trochlear nerve injuries can include:

1. Double vision (diplopia)
2. Drooping of the eyelid (ptosis)
3. Difficulty moving the eyes (oculomotor dysfunction)
4. Pain or discomfort in the eye or orbit
5. Redness or swelling of the conjunctiva (the thin membrane covering the white part of the eye)
6. Difficulty closing the eyelid completely
7. Sensitivity to light (photophobia)
8. Blurred vision
9. Nausea and vomiting
10. Headache

Diagnosis of trochlear nerve injuries is typically made through a combination of physical examination, imaging studies such as CT or MRI scans, and specialized tests such as electromyography (EMG) or visual evoked potentials (VEP).

Treatment of trochlear nerve injuries depends on the underlying cause and severity of the injury. In some cases, surgery may be necessary to repair the nerve or relieve compression on the nerve. Physical therapy and exercises may also be recommended to help restore strength and range of motion in the affected eye. Medications such as anticholinergics or anti-inflammatory drugs may be prescribed to relieve symptoms such as dryness, redness, or pain. In cases where the injury is caused by a more serious condition such as a tumor or aneurysm, treatment of the underlying condition may be necessary before addressing the nerve injury.

Glomus jugulare tumors are thought to arise from abnormal growth of cells called glomus cells, which are found in the walls of blood vessels throughout the body. These cells play a role in regulating blood pressure by producing certain hormones and chemicals that help to constrict or dilate blood vessels.

Glomus jugulare tumors can cause a variety of symptoms depending on their size and location, including:

1. Swelling in the neck or face
2. Pain in the neck or face
3. Difficulty swallowing
4. Numbness or weakness in the face
5. Vision problems

If a glomus jugulare tumor is suspected, a doctor may perform several tests to confirm the diagnosis and determine the best course of treatment. These tests may include:

1. Ultrasound: This non-invasive imaging test uses sound waves to create pictures of the blood vessels in the neck.
2. Computed tomography (CT) scan: This test uses X-rays and computer technology to create detailed images of the neck and head.
3. Magnetic resonance imaging (MRI): This test uses a strong magnetic field and radio waves to create detailed images of the blood vessels in the neck.
4. Biopsy: In this test, a small sample of tissue is removed from the jugular vein and examined under a microscope to confirm the presence of a glomus jugulare tumor.

If a glomus jugulare tumor is diagnosed, treatment may involve one or more of the following:

1. Surgery to remove the tumor
2. Embolization, in which a small catheter is inserted into the jugular vein and a substance is injected to block the blood flow to the tumor
3. Radiation therapy, in which high-energy rays are used to kill the cancer cells
4. Chemotherapy, in which drugs are used to kill the cancer cells.

It's important to note that each case is unique and the treatment plan will be tailored to the individual patient's needs. The best course of treatment will depend on the location, size, and aggressiveness of the tumor, as well as other factors such as the patient's overall health and medical history.

Types of Peripheral Nerve Injuries:

1. Traumatic Nerve Injury: This type of injury occurs due to direct trauma to the nerve, such as a blow or a crush injury.
2. Compression Neuropathy: This type of injury occurs when a nerve is compressed or pinched, leading to damage or disruption of the nerve signal.
3. Stretch Injury: This type of injury occurs when a nerve is stretched or overstretched, leading to damage or disruption of the nerve signal.
4. Entrapment Neuropathy: This type of injury occurs when a nerve is compressed or trapped between two structures, leading to damage or disruption of the nerve signal.

Symptoms of Peripheral Nerve Injuries:

1. Weakness or paralysis of specific muscle groups
2. Numbness or tingling in the affected area
3. Pain or burning sensation in the affected area
4. Difficulty with balance and coordination
5. Abnormal reflexes
6. Incontinence or other bladder or bowel problems

Causes of Peripheral Nerve Injuries:

1. Trauma, such as a car accident or fall
2. Sports injuries
3. Repetitive strain injuries, such as those caused by repetitive motions in the workplace or during sports activities
4. Compression or entrapment of nerves, such as carpal tunnel syndrome or tarsal tunnel syndrome
5. Infections, such as Lyme disease or diphtheria
6. Tumors or cysts that compress or damage nerves
7. Vitamin deficiencies, such as vitamin B12 deficiency
8. Autoimmune disorders, such as rheumatoid arthritis or lupus
9. Toxins, such as heavy metals or certain chemicals

Treatment of Peripheral Nerve Injuries:

1. Physical therapy to improve strength and range of motion
2. Medications to manage pain and inflammation
3. Surgery to release compressed nerves or repair damaged nerves
4. Electrical stimulation therapy to promote nerve regeneration
5. Platelet-rich plasma (PRP) therapy to stimulate healing
6. Stem cell therapy to promote nerve regeneration
7. Injection of botulinum toxin to relieve pain and reduce muscle spasticity
8. Orthotics or assistive devices to improve mobility and function

It is important to seek medical attention if you experience any symptoms of a peripheral nerve injury, as early diagnosis and treatment can help prevent long-term damage and improve outcomes.

Causes:

1. Refractive errors: Diplopia can be caused by refractive errors such as myopia (nearsightedness), hyperopia (farsightedness), astigmatism, or presbyopia (age-related loss of near vision).
2. Eye alignment problems: Disorders such as strabismus (crossed eyes) or esotropia (eyes turned inward) can cause diplopia.
3. Cataracts: A cataract can cause diplopia due to the clouding of the lens in one or both eyes.
4. Glaucoma: Diplopia can be a symptom of glaucoma, a group of eye conditions that damage the optic nerve.
5. Retinal detachment: A retinal detachment can cause diplopia due to the separation of the retina from the underlying tissue.
6. Brain injuries or disorders: Diplopia can be a result of brain injuries or disorders such as stroke, traumatic brain injury, or multiple sclerosis.
7. Medications: Certain medications such as antidepressants, anti-seizure drugs, and chemotherapy drugs can cause diplopia as a side effect.

Diagnosis:

To diagnose diplopia, an eye examination is necessary. The doctor may perform a cover test to determine the type of diplopia and rule out other conditions. Imaging tests such as ultrasound or MRI may also be ordered to examine the eye and surrounding tissues.

Treatment:

The treatment of diplopia depends on the underlying cause. In some cases, glasses or contact lenses can correct refractive errors and alleviate symptoms. Surgery may be necessary for eye alignment problems such as strabismus or cataracts. In cases where the condition is caused by a brain disorder or injury, treatment of the underlying condition can resolve diplopia.

Prognosis:

The prognosis for diplopia varies depending on the underlying cause. In some cases, the condition may resolve on its own or with simple correction such as glasses or contact lenses. In other cases, surgery or treatment of an underlying condition may be necessary to resolve diplopia. In rare cases, the condition can lead to complications such as amblyopia (lazy eye) or vision loss if left untreated.

Prevention:

Preventing diplopia is not always possible, but early detection and treatment of underlying conditions can help prevent complications and improve outcomes. Regular eye exams and monitoring of vision can also help detect diplopia early on. In some cases, prism lenses or glasses with a specific prescription may be recommended to alleviate symptoms and prevent progression of the condition.

In conclusion, diplopia is a common condition that can have various causes and underlying mechanisms. Early diagnosis and treatment are crucial to prevent complications and improve outcomes. Regular eye exams and monitoring of vision can help detect diplopia early on, and in some cases, simple correction such as glasses or contact lenses may be sufficient to resolve the condition. In other cases, surgery or treatment of an underlying condition may be necessary. With appropriate management, most people with diplopia can achieve good visual acuity and quality of life.

Types of Glossopharyngeal Nerve Diseases:

1. Glossopharyngeal Neuralgia: This is a condition characterized by recurring episodes of sudden, severe pain in the tongue, throat, and ears. The pain can be triggered by coughing, swallowing, or other movements.
2. Glossopharyngeal Nerve Palsy: This is a condition where the glossopharyngeal nerve is damaged, leading to weakness or paralysis of the tongue and other muscles in the throat and mouth.
3. Glossopharyngeal Fibrillation: This is a condition characterized by rapid, involuntary contractions of the muscles in the throat, which can cause difficulty swallowing and other symptoms.

Causes of Glossopharyngeal Nerve Diseases:

1. Trauma to the head or neck
2. Viral infections such as herpes zoster (shingles) or Lyme disease
3. Bacterial infections such as meningitis or abscesses
4. Tumors or cysts in the throat or brain
5. Inflammatory conditions such as rheumatoid arthritis or sarcoidosis
6. Genetic disorders such as Charcot-Marie-Tooth disease or other inherited neurological conditions

Symptoms of Glossopharyngeal Nerve Diseases:

1. Pain in the tongue, throat, and ears
2. Weakness or paralysis of the tongue and other muscles in the throat and mouth
3. Difficulty swallowing (dysphagia)
4. Hoarseness or other changes in voice quality
5. Loss of taste sensation
6. Ear pain or hearing loss
7. Fatigue, weakness, or numbness in the face and neck
8. Involuntary movements of the tongue, lips, or jaw (tics)
9. Difficulty articulating speech
10. Coughing or choking on food or liquids.

Diagnosis of Glossopharyngeal Nerve Diseases:

1. Medical history and physical examination
2. Imaging studies such as CT or MRI scans
3. Electromyography (EMG) to test the function of muscles in the throat and face
4. Nerve conduction studies (NCS) to evaluate the function of nerves
5. Biopsy of tissue samples from the throat or neck to rule out other conditions.

Treatment for Glossopharyngeal Nerve Diseases:

1. Medications such as antiviral, antibiotic, or anti-inflammatory drugs
2. Surgery to remove tumors or treat nerve damage
3. Physical therapy to improve swallowing and speaking difficulties
4. Speech therapy to improve communication skills
5. Lifestyle changes such as avoiding irritants, maintaining good oral hygiene, and eating a balanced diet.

Prognosis for Glossopharyngeal Nerve Diseases:

The prognosis for glossopharyngeal nerve diseases varies depending on the underlying cause and severity of the condition. In general, with proper treatment and management, many patients can experience significant improvement in their symptoms and quality of life. However, some patients may experience persistent or recurrent symptoms, and in rare cases, the condition can be life-threatening. Early diagnosis and treatment are essential to achieve the best possible outcomes.

The exact cause of neurilemmoma is not known, but it is believed to be related to genetic mutations that occur during fetal development. Some cases have been associated with neurofibromatosis type 2, a genetic disorder that affects the growth and development of nerve tissue.

Neurilemmoma typically manifests as a slow-growing mass or lump in the affected area. Symptoms can include pain, numbness, tingling, or weakness in the affected limb or organ, depending on the location of the tumor. In some cases, neurilemmoma can cause hormonal imbalances or disrupt normal nerve function.

Diagnosis of neurilemmoma usually involves a combination of physical examination, imaging studies such as MRI or CT scans, and a biopsy to confirm the presence of malignant cells. Treatment options for neurilemmoma include surgical removal of the tumor, radiation therapy, and in some cases, observation with periodic monitoring. The prognosis for patients with neurilemmoma is generally good if the tumor is removed completely, but recurrence is possible in some cases.

There are several ways in which glossopharyngeal nerve injuries can occur, including:

1. Trauma to the head or neck: A blow to the head or neck can cause damage to the glossopharyngeal nerve, leading to injury.
2. Surgery: Certain surgical procedures, such as thyroidectomy or tonsillectomy, can cause temporary or permanent damage to the glossopharyngeal nerve.
3. Infections: Certain infections, such as meningitis or abscesses, can spread to the glossopharyngeal nerve and cause injury.
4. Tumors: Tumors in the head or neck can compress or damage the glossopharyngeal nerve, leading to injury.
5. Ischemia: Reduced blood flow to the brain or neck can cause injury to the glossopharyngeal nerve.

Symptoms of glossopharyngeal nerve injuries can vary depending on the location and severity of the injury, but may include:

1. Difficulty swallowing (dysphagia)
2. Hoarseness or changes in voice
3. Weakness or paralysis of the tongue
4. Numbness or tingling in the throat or tongue
5. Pain in the throat or tongue

Treatment for glossopharyngeal nerve injuries depends on the underlying cause and severity of the injury, but may include:

1. Medications to relieve pain and inflammation
2. Physical therapy to improve swallowing and speech difficulties
3. Surgery to repair or remove damaged tissue
4. Speech therapy to improve communication skills
5. Lifestyle changes, such as avoiding heavy lifting or bending, to reduce pressure on the nerve.

Synonyms: acoustic neuroma, vestibular schwannoma

Previous term: Necropsy Next term: Neurodegeneration

The vagus nerve is a complex nerve that runs from the brain to various parts of the body, including the throat, heart, lungs, and digestive system. It plays a crucial role in regulating various bodily functions such as breathing, swallowing, and heart rate. Injuries to the vagus nerve can occur due to trauma, surgery, or other medical conditions.

The symptoms of vagus nerve injuries can vary depending on the severity and location of the injury. Some common symptoms include:

* Difficulty speaking or swallowing
* Slurred speech or hoarseness
* Weakness or paralysis of the vocal cords
* Difficulty breathing or shortness of breath
* Chest pain or tightness
* Heart rate variability or arrhythmias
* Digestive problems such as nausea, vomiting, or diarrhea

Vagus nerve injuries can be difficult to diagnose, as the symptoms can be subtle and may resemble other conditions. A thorough medical evaluation, including neurological examination and imaging studies, is necessary for accurate diagnosis. Treatment of vagus nerve injuries depends on the underlying cause and severity of the injury. In some cases, surgery may be required to repair or replace the damaged nerve. Physical therapy and speech therapy may also be helpful in regaining function and improving quality of life.

The exact cause of CBTs is not fully understood, but they are thought to be associated with genetic mutations and may be more common in people with a family history of similar tumors. The diagnosis of a carotid body tumor is typically made using imaging tests such as ultrasound, CT or MRI scans, and a biopsy may be performed to confirm the diagnosis.

Treatment for CBTs usually involves surgical removal of the tumor, and in some cases, radiation therapy may also be recommended to reduce the risk of recurrence. The prognosis for patients with CBTs is generally good, but the tumors can recur in some cases.

Preventive measures: There are no specific preventive measures known to prevent carotid body tumors, but early detection and treatment can improve outcomes. Regular neck checks and imaging tests may be recommended for individuals with a family history of these tumors or those who experience symptoms.

Current research: Researchers are working to better understand the causes of CBTs and to develop new treatments that can improve outcomes for patients with these tumors. Studies are ongoing to investigate the genetic mutations that contribute to the development of CBTs and to identify potential targets for therapy. Additionally, researchers are exploring the use of minimally invasive surgical techniques and radiotherapy to treat CBTs.

In summary, carotid body tumors are rare but potentially symptomatic vascular tumors that can be diagnosed and treated with surgery and/or radiation therapy. Early detection and treatment can improve outcomes, and ongoing research is focused on understanding the causes of these tumors and developing new treatments.

Duane's retraction syndrome is caused by an abnormality in the nerves that control eyelid movement. The condition may be treated with surgery to correct any underlying abnormalities or to improve the functioning of the affected eye(s).

The symptoms of meningeal carcinomatosis can vary depending on the location and extent of the tumor, but may include headaches, seizures, weakness or numbness in the arms or legs, and changes in personality or mental status. The diagnosis is typically made by a combination of physical examination, imaging studies such as CT or MRI scans, and laboratory tests to detect the presence of cancer cells in the cerebrospinal fluid (CSF).

Treatment of meningeal carcinomatosis depends on the underlying cause and the extent of the tumor. Treatment options may include surgery, radiation therapy, and chemotherapy, as well as supportive care to manage symptoms such as pain, seizures, and infection. The prognosis for meningeal carcinomatosis is generally poor, with a five-year survival rate of less than 10%.

The hypoglossal nerve is a cranial nerve that controls the movement of the tongue and other muscles in the throat. Hypoglossal nerve diseases refer to conditions that affect the functioning of this nerve, leading to symptoms such as difficulty swallowing, weakness or paralysis of the tongue, and speech difficulties.

Some examples of hypoglossal nerve diseases include:

1. Hypoglossal neuritis: This is an inflammation of the hypoglossal nerve, which can be caused by viral infections, head injuries, or other conditions.
2. Hypoglossal nerve palsy: This is a condition where the hypoglossal nerve is damaged or compressed, leading to weakness or paralysis of the tongue and other muscles in the throat.
3. Congenital hypoglossal nerve defects: These are birth defects that affect the development of the hypoglossal nerve, leading to a range of symptoms including difficulty swallowing and speech difficulties.
4. Trauma to the hypoglossal nerve: This can occur due to injury or trauma to the neck or head, leading to weakness or paralysis of the tongue and other muscles in the throat.
5. Tumors or cysts affecting the hypoglossal nerve: These can cause compression or damage to the nerve, leading to symptoms such as difficulty swallowing, speech difficulties, and weakness or paralysis of the tongue.

Hypoglossal nerve diseases can be diagnosed through a range of tests, including electromyography (EMG), nerve conduction studies (NCS), and imaging studies such as MRI or CT scans. Treatment depends on the underlying cause of the condition and may include physical therapy, medication, or surgery.

There are different types of hyperostosis, including:

1. Hyperostosis fibrosa: This is a condition where there is excessive bone growth in the thickening of the cortical bone, leading to a hard and firm consistency. It can occur in various parts of the body, such as the skull, spine, or long bones.
2. Hyperostosis iritis: This is a condition where there is excessive bone growth in the iris of the eye, leading to symptoms such as vision loss, pain, and light sensitivity.
3. Hyperostosis mediastinitis: This is a rare condition where there is excessive bone growth in the mediastinum, a region between the lungs and the spine. It can cause compression of nearby structures and lead to symptoms such as difficulty swallowing, chest pain, and shortness of breath.
4. Hyperostosis of the sacrum: This is a condition where there is excessive bone growth in the sacrum, a triangular bone at the base of the spine. It can cause compression of nearby structures and lead to symptoms such as lower back pain, sciatica, and difficulty walking.

Hyperostosis can be diagnosed through imaging tests such as X-rays, CT scans, or MRI scans. Treatment options depend on the underlying cause and may include medications to manage symptoms, physical therapy, or surgery to remove excess bone growth.

Aseptic meningitis can cause a range of symptoms, including headache, fever, stiff neck, nausea and vomiting, sensitivity to light, and confusion. In severe cases, it can lead to brain damage, seizures, and even death.

Aseptic meningitis is diagnosed through a combination of physical examination, medical history, laboratory tests (such as blood cultures and cerebrospinal fluid analysis), and imaging studies (such as CT or MRI scans). Treatment typically involves supportive care, such as intravenous fluids and pain management, as well as addressing any underlying causes. In some cases, antibiotics may be prescribed if a bacterial infection is suspected.

Aseptic meningitis can affect anyone, but it is more common in certain groups, such as children under the age of 5 and people with weakened immune systems. It is important to seek medical attention immediately if symptoms persist or worsen over time.

Recurrent laryngeal nerve injuries refer to damage or trauma to the recurrent laryngeal nerve, which is a branch of the vagus nerve that supplies motor and sensory functions to the larynx (voice box) and other structures in the neck and throat. These injuries can occur due to various causes such as surgery, trauma, or degenerative conditions.

Types of Recurrent Laryngeal Nerve Injuries:

There are several types of recurrent laryngeal nerve injuries, including:

1. Traumatic injury: This type of injury occurs due to direct blows or penetrating wounds to the neck or throat.
2. Ischemic injury: This type of injury occurs due to reduced blood flow to the nerve, often due to atherosclerosis (narrowing of the blood vessels) or other conditions that affect blood flow.
3. Neuritis: This type of injury occurs due to inflammation of the nerve, often due to viral infections such as herpes zoster (shingles).
4. Tumors: Benign or malignant tumors in the neck or throat can compress or damage the recurrent laryngeal nerve.
5. Surgical injury: Recurrent laryngeal nerve injuries can occur during surgical procedures such as thyroid or parathyroid surgery, or laryngotomy (surgery on the voice box).

Symptoms of Recurrent Laryngeal Nerve Injuries:

The symptoms of recurrent laryngeal nerve injuries can vary depending on the severity and location of the injury. Common symptoms include:

1. Hoarseness or weakness of the voice
2. Difficulty swallowing (dysphagia)
3. Pain in the neck, throat, or ear
4. Numbness or tingling sensations in the neck or face
5. Weakness or paralysis of the vocal cords
6. Inability to speak or vocalize
7. Breathing difficulties

Diagnosis and Treatment of Recurrent Laryngeal Nerve Injuries:

To diagnose a recurrent laryngeal nerve injury, a thorough medical history and physical examination are essential. Imaging studies such as MRI or CT scans may also be ordered to confirm the presence and extent of the injury. Electromyography (EMG) and nerve conduction studies (NCS) may also be performed to assess the function of the nerve.

Treatment of recurrent laryngeal nerve injuries depends on the underlying cause and severity of the injury. Some common treatment options include:

1. Supportive care: Patients with mild symptoms may require only supportive care, such as voice therapy or speech therapy to improve communication.
2. Medications: Anti-inflammatory medications or steroids may be prescribed to reduce swelling and inflammation.
3. Surgery: In some cases, surgical intervention may be necessary to repair the damaged nerve or remove any compressive lesions.
4. Botulinum toxin injections: Botulinum toxin injections can be used to relax the vocal cord muscles and improve voice quality.
5. Thyroid hormone replacement: Patients with hypothyroidism may require thyroid hormone replacement therapy to improve vocal cord function.
6. Laryngeal framework surgery: This type of surgery is used to correct any structural abnormalities in the larynx that may be contributing to the nerve injury.
7. Vocal fold injection: Injecting material into the vocal folds can help to improve voice quality and reduce symptoms.
8. Speech therapy: Patients with persistent symptoms may require speech therapy to improve communication and address any swallowing difficulties.

Conclusion:

Recurrent laryngeal nerve injuries can have a significant impact on an individual's quality of life, causing a range of symptoms that affect communication, breathing, and swallowing. Prompt diagnosis and appropriate treatment are essential to prevent long-term damage and improve outcomes. While treatment options vary depending on the underlying cause and severity of the injury, surgical interventions, botulinum toxin injections, and speech therapy may be effective in managing symptoms and improving voice quality.

Hemifacial spasm is a relatively rare movement disorder that affects one side of the face. It is characterized by involuntary muscle contractions and twitching on half of the face, which can be quite distressing for those who experience it. While there are several possible causes, including nerve compression or brain tumors, the exact cause is often difficult to determine.

One of the most common symptoms of HFS is muscle spasms and twitching on one side of the face, which can be quite pronounced and unpredictable. These spasms can occur in any of the muscles on the affected side, including those around the eyes, mouth, and jaw. In some cases, these spasms can also affect the eyelids, causing them to droop or close involuntarily.

The exact cause of hemifacial spasm is not always clear, but it is believed to be related to nerve compression or irritation of the facial nerve. This nerve runs from the brain down through the face and controls many of the muscles in the face, including those involved in eyelid movement and facial expressions. When this nerve is compressed or irritated, it can cause the muscles on one side of the face to spasm and twitch involuntarily.

There are several possible causes of HFS, including:

* Compression of the facial nerve by a blood vessel or tumor
* Trauma to the face or head
* Inflammatory conditions such as multiple sclerosis or sarcoidosis
* Brain tumors or cysts
* Stroke or other forms of brain damage

Treatment for hemifacial spasm usually involves a combination of medications and other therapies. Botulinum toxin injections are often used to relax the affected muscles and reduce spasms. Medications such as anticonvulsants, muscle relaxants, or anti-anxiety drugs may also be prescribed to help manage symptoms. In some cases, surgery may be necessary to relieve compression on the facial nerve.

In addition to these medical treatments, there are also several self-care techniques that can help manage hemifacial spasm. These include:

* Avoiding triggers such as stress or fatigue
* Applying warm compresses to the affected side of the face
* Practicing relaxation techniques such as deep breathing or meditation
* Using eye exercises to strengthen the muscles around the eyes and improve eyelid function.

It is important to seek medical attention if you are experiencing symptoms of hemifacial spasm, as early diagnosis and treatment can help prevent complications and improve outcomes. With proper management, many people with HFS are able to effectively manage their symptoms and lead normal lives.

Examples of Skull Neoplasms include:

1. Meningioma: A benign tumor that arises from the meninges, the protective covering of the brain and spinal cord.
2. Acoustic neuroma: A benign tumor that grows on the nerve that connects the inner ear to the brain.
3. Pineal parenchymal tumors: Tumors that arise in the pineal gland, a small endocrine gland located in the brain.
4. Craniopharyngiomas: Benign tumors that arise near the pituitary gland, which regulates hormone production.
5. Medulloblastoma: A malignant tumor that arises in the cerebellum, a part of the brain that controls movement and coordination.
6. Germ cell tumors: Tumors that arise from immature cells that form in the embryo. These can be benign or malignant.
7. PNETs (primitive neuroectodermal tumors): Malignant tumors that arise from early forms of nerve cells.
8. Astrocytomas: Tumors that arise from the supportive tissue of the brain called astrocytes. These can be benign or malignant.
9. Oligodendrogliomas: Tumors that arise from the supportive tissue of the brain called oligodendrocytes. These can be benign or malignant.
10. Melanotic neuroectodermal tumors: Rare, malignant tumors that contain pigmented cells.

Symptoms of Abducens Nerve Injury:

The symptoms of abducens nerve injury can vary depending on the severity and location of the injury. Some common symptoms include:

* Double vision or blurred vision
* Difficulty moving the eyes inward or outward
* Difficulty focusing the eyes
* Weakness or paralysis of the eyelid
* Headaches or eye strain
* Dizziness or nausea

Causes of Abducens Nerve Injury:

Abducens nerve injury can be caused by a variety of factors, including:

* Trauma to the head or face
* Tumors or cysts in the brain or near the nerve
* Stroke or other conditions that affect blood flow to the brain
* Infections such as meningitis or encephalitis
* Viral or bacterial infections that spread to the nerve
* Poorly positioned glasses or contact lenses
* Trauma from surgery or other medical procedures

Diagnosis and Treatment of Abducens Nerve Injury:

The diagnosis of abducens nerve injury is typically made through a combination of physical examination, imaging tests such as CT or MRI scans, and other diagnostic tests. Treatment depends on the underlying cause of the injury and may include:

* Glasses or contact lenses to correct vision problems
* Eye exercises to strengthen the affected eye muscles
* Prism lenses to help align the eyes properly
* Surgery to repair damaged tissue or relieve pressure on the nerve
* Physical therapy to improve eye movement and coordination
* Medications to manage pain, swelling, or inflammation

Prognosis for Abducens Nerve Injury:

The prognosis for abducens nerve injury depends on the underlying cause of the injury and the promptness and effectiveness of treatment. In general, the earlier treatment is received, the better the outcome. If the injury is caused by a treatable condition, such as a tumor or infection, the prognosis is generally good if the condition can be successfully treated. However, if the injury is caused by a more severe or permanent condition, such as a stroke, the prognosis may be poorer.

Prevention of Abducens Nerve Injury:

There are several steps you can take to help prevent abducens nerve injury, including:

* Wearing protective eyewear during activities that could potentially cause eye injury
* Avoiding head trauma by taking precautions such as wearing seatbelts and helmets
* Keeping the head and neck in a neutral position during sleep to avoid putting pressure on the nerve
* Managing any underlying medical conditions, such as diabetes or high blood pressure, which can increase the risk of nerve damage
* Avoiding smoking and excessive alcohol consumption, which can damage the nerves

Living with Abducens Nerve Injury:

Living with abducens nerve injury can be challenging, as it can affect your ability to move your eyes and potentially impact your vision. However, there are several strategies that can help you adapt to the condition and improve your quality of life. These may include:

* Using prisms or other visual aids to help align your vision
* Compensating for any double vision by using eye exercises or other techniques
* Adjusting your daily activities to accommodate any limitations in your vision
* Seeking support from family, friends, and healthcare professionals to cope with the emotional impact of the condition.

Meningioma can occur in various locations within the brain, including the cerebrum, cerebellum, brainstem, and spinal cord. The most common type of meningioma is the meningothelial meningioma, which arises from the arachnoid membrane, one of the three layers of the meninges. Other types of meningioma include the dural-based meningioma, which originates from the dura mater, and the fibrous-cap meningioma, which is characterized by a fibrous cap covering the tumor.

The symptoms of meningioma can vary depending on the location and size of the tumor, but they often include headaches, seizures, weakness or numbness in the arms or legs, and changes in vision, memory, or cognitive function. As the tumor grows, it can compress the brain tissue and cause damage to the surrounding structures, leading to more severe symptoms such as difficulty speaking, walking, or controlling movement.

The diagnosis of meningioma typically involves a combination of imaging studies such as MRI or CT scans, and tissue sampling through biopsy or surgery. Treatment options for meningioma depend on the size, location, and aggressiveness of the tumor, but may include surgery, radiation therapy, and chemotherapy. Overall, the prognosis for meningioma is generally good, with many patients experiencing a good outcome after treatment. However, some types of meningioma can be more aggressive and difficult to treat, and the tumor may recur in some cases.

Example sentences:

1. The patient experienced a spasm in their leg while running, causing them to stumble and fall.
2. The doctor diagnosed the patient with muscle spasms caused by dehydration and recommended increased fluids and stretching exercises.
3. The athlete suffered from frequent leg spasms during their training, which affected their performance and required regular massage therapy to relieve the discomfort.

Vertigo can cause a range of symptoms, including:

* A feeling of spinning or swaying
* Dizziness or lightheadedness
* Blurred vision
* Nausea and vomiting
* Abnormal eye movements
* Unsteadiness or loss of balance

To diagnose vertigo, a healthcare professional will typically conduct a physical examination and ask questions about the patient's symptoms and medical history. They may also perform tests such as the head impulse test or the electronystagmography (ENG) test to assess the function of the inner ear and balance systems.

Treatment for vertigo depends on the underlying cause, but may include medications such as anticholinergics, antihistamines, or benzodiazepines, as well as vestibular rehabilitation therapy (VRT) to help the body adapt to the balance problems. In some cases, surgery may be necessary to treat the underlying cause of vertigo.

In summary, vertigo is a symptom characterized by a false sense of spinning or movement of the surroundings, and can be caused by various conditions affecting the inner ear, brain, or nervous system. Diagnosis and treatment depend on the underlying cause, but may include medications, VRT, and in some cases, surgery.

The symptoms of meningeal neoplasms vary depending on the location, size, and type of tumor. Common symptoms include headaches, seizures, weakness or numbness in the arms or legs, and changes in vision, memory, or behavior. As the tumor grows, it can compress or displaces the brain tissue, leading to increased intracranial pressure and potentially life-threatening complications.

There are several different types of meningeal neoplasms, including:

1. Meningioma: This is the most common type of meningeal neoplasm, accounting for about 75% of all cases. Meningiomas are usually benign and grow slowly, but they can sometimes be malignant.
2. Metastatic tumors: These are tumors that have spread to the meninges from another part of the body, such as the lung or breast.
3. Lymphoma: This is a type of cancer that affects the immune system and can spread to the meninges.
4. Melanotic neuroectodermal tumors (MNTs): These are rare, malignant tumors that usually occur in children and young adults.
5. Hemangiopericytic hyperplasia: This is a rare, benign condition characterized by an overgrowth of blood vessels in the meninges.

The diagnosis of meningeal neoplasms is based on a combination of clinical symptoms, physical examination findings, and imaging studies such as CT or MRI scans. A biopsy may be performed to confirm the diagnosis and determine the type of tumor.

Treatment options for meningeal neoplasms depend on the type, size, and location of the tumor, as well as the patient's overall health. Surgery is often the first line of treatment, and may involve removing as much of the tumor as possible or using a laser to ablate (destroy) the tumor cells. Radiation therapy and chemotherapy may also be used in combination with surgery to treat malignant meningeal neoplasms.

Prognosis for meningeal neoplasms varies depending on the type of tumor and the patient's overall health. In general, early diagnosis and treatment improve the prognosis, while later-stage tumors may have a poorer outcome.

Types of Optic Nerve Injuries:

1. Traumatic optic neuropathy: This type of injury is caused by direct damage to the optic nerve as a result of trauma, such as a car accident or sports injury.
2. Ischemic optic neuropathy: This type of injury is caused by a lack of blood flow to the optic nerve, which can lead to cell death and vision loss.
3. Inflammatory optic neuropathy: This type of injury is caused by inflammation of the optic nerve, which can be caused by conditions such as multiple sclerosis or sarcoidosis.
4. Tumor-induced optic neuropathy: This type of injury is caused by a tumor that compresses or damages the optic nerve.
5. Congenital optic nerve disorders: These are present at birth and can cause vision loss or blindness. Examples include optic nerve hypoplasia and coloboma.

Symptoms of Optic Nerve Injuries:

* Blurred vision or double vision
* Loss of peripheral vision
* Difficulty seeing in dim lighting
* Pain or discomfort in the eye or head
* Redness or swelling of the eye

Diagnosis and Treatment of Optic Nerve Injuries:

Diagnosis is typically made through a combination of physical examination, imaging tests such as MRI or CT scans, and visual field testing. Treatment depends on the underlying cause of the injury, but may include medication, surgery, or vision rehabilitation. In some cases, vision loss may be permanent, but early diagnosis and treatment can help to minimize the extent of the damage.

Prognosis for Optic Nerve Injuries:

The prognosis for optic nerve injuries varies depending on the underlying cause and severity of the injury. In some cases, vision may be partially or fully restored with treatment. However, in other cases, vision loss may be permanent. It is important to seek medical attention immediately if any symptoms of an optic nerve injury are present, as early diagnosis and treatment can improve outcomes.

There are several types of polyradiculoneuropathy, each with its own set of causes and characteristics:

1. Polyneuropathy: This is the most common type of polyradiculoneuropathy and affects multiple nerves throughout the body. It can be caused by a variety of factors, such as diabetes, vitamin deficiencies, alcoholism, and certain medications.
2. Mononeuritis multiplex: This is a condition in which there is damage to multiple nerves that innervate a specific area of the body, such as the legs or arms. It can be caused by various factors, including diabetes, autoimmune disorders, and certain medications.
3. Radiculoneuropathy: This type of polyradiculoneuropathy affects the nerves that originate from the spinal cord and extend to other parts of the body. It can be caused by compression or inflammation of the nerve roots, such as in the case of herniated discs or spinal stenosis.
4. Autonomic neuropathy: This type of polyradiculoneuropathy affects the nerves that control involuntary functions, such as heart rate, blood pressure, and digestion. It can be caused by a variety of factors, including diabetes, vitamin deficiencies, and certain medications.

The symptoms of polyradiculoneuropathy can vary depending on the specific type and severity of the condition. Common symptoms include:

* Weakness or numbness in the affected areas
* Pain or discomfort in the affected areas
* Difficulty walking or maintaining balance
* Difficulty with fine motor skills, such as buttoning a shirt or tying shoelaces
* Digestive problems, such as constipation or diarrhea
* Urinary incontinence or retention

The diagnosis of polyradiculoneuropathy is typically made based on a combination of physical examination findings, medical history, and results of diagnostic tests such as nerve conduction studies or electromyography. Treatment options for polyradiculoneuropathy depend on the underlying cause of the condition, but may include:

* Medications to manage pain or inflammation
* Physical therapy to improve strength and coordination
* Lifestyle modifications, such as quitting smoking or losing weight, to reduce pressure on the nerves
* Surgery to relieve compression or repair damaged nerves

In some cases, polyradiculoneuropathy may be a symptom of an underlying condition that can be treated or managed with medication or other therapies. It is important to seek medical attention if you experience any symptoms of polyradiculoneuropathy to receive an accurate diagnosis and appropriate treatment.

The main symptoms of Horner syndrome include:

1. Pain and numbness in the face and arm on one side of the body
2. Weakness or paralysis of the muscles on one side of the face, arm, and hand
3. Difficulty swallowing
4. Reduced sweating on one side of the body
5. Increased heart rate and blood pressure
6. Narrowing of the pupil (anisocoria)
7. Dilation of the unaffected pupil (paralysis of the parasympathetic nervous system)
8. Decreased reflexes
9. Loss of sensation in the skin over the chest and abdomen
10. Pale or clammy skin on one side of the body

The symptoms of Horner syndrome can be caused by a variety of factors, including:

1. Trauma to the thoracolumbar spine
2. Injury or tumor in the brainstem or spinal cord
3. Aneurysm or arteriovenous malformation (AVM) in the neck or chest
4. Multiple sclerosis, amyotrophic lateral sclerosis (ALS), or other neurodegenerative diseases
5. Inflammatory conditions such as sarcoidosis or tuberculosis
6. Infections such as meningitis or abscesses
7. Vasospasm or thrombosis of the blood vessels in the neck or chest.

The diagnosis of Horner syndrome is based on a combination of clinical findings, neuroimaging studies (such as MRI or CT scans), and laboratory tests to rule out other causes of the symptoms. Treatment of the condition depends on the underlying cause and may include surgery, medication, or other interventions. In some cases, Horner syndrome may be a sign of a more serious underlying condition that requires prompt medical attention.

The symptoms of Herpes Zoster Oticus include:

* Painful shingles rash on the face, ears, and neck
* Hearing loss or tinnitus (ringing in the ears)
* Vertigo or dizziness
* Fatigue
* Headache
* Numbness or paralysis of the facial muscles
* Redness and swelling of the eye and eyelid

The diagnosis of Herpes Zoster Oticus is based on a combination of physical examination, medical history, and laboratory tests. The condition is usually treated with antiviral medications, corticosteroids, and pain relief medication. In severe cases, surgical intervention may be necessary to relieve pressure on the nerve or to repair damaged tissue.

Complications of Herpes Zoster Oticus can include:

* Meningitis (inflammation of the membranes that cover the brain and spinal cord)
* Encephalitis (inflammation of the brain)
* Hearing loss or deafness
* Balance and coordination problems
* Persistent facial weakness or paralysis
* Eye inflammation

Prevention of Herpes Zoster Oticus includes avoiding exposure to the varicella-zoster virus, getting vaccinated against chickenpox, and taking antiviral medications to suppress the virus if exposed. Good hygiene practices, such as frequent handwashing, can also help prevent the spread of the virus.

In summary, Herpes Zoster Oticus is a viral infection that affects the facial nerve near the inner ear and the skin around the ears, causing a painful rash, hearing loss, vertigo, and other symptoms. The condition can be treated with antiviral medications and surgical intervention, and prevention includes avoiding exposure to the virus and practicing good hygiene practices.

The main features of NF2 include:

1. Tumor growth: NF2 patients develop meningiomas or schwannomas, which are benign tumors that can grow and compress nearby nerves.
2. Vision loss: The compression of optic nerves by tumors can lead to vision loss or blindness.
3. Hearing loss: Tumors can also affect the auditory nerve, leading to hearing loss or deafness.
4. Balance and coordination problems: Tumors can cause balance and coordination problems due to their effects on the cranial nerves.
5. Cognitive impairment: NF2 patients may experience cognitive impairment, including memory loss, confusion, and difficulty with concentration.
6. Seizures: Some patients with NF2 may experience seizures as a result of tumor growth or other factors.
7. Pain: Tumors can cause pain, either due to their size or location.
8. Headaches: NF2 patients may experience frequent headaches due to the pressure of tumors on surrounding nerves and brain tissue.
9. Endocrine dysfunction: Some patients with NF2 may experience endocrine dysfunction, including thyroid problems or growth hormone deficiency.
10. Increased risk of other cancers: NF2 patients have an increased risk of developing other types of cancer, particularly malignant melanoma.

The diagnosis of NF2 is based on a combination of clinical features, imaging studies (such as MRI), and genetic testing. Treatment options for NF2 include observation, surgery, radiation therapy, and chemotherapy, depending on the size and location of the tumors and the severity of symptoms.

Causes and risk factors:

The exact cause of brain stem neoplasms is not fully understood, but they can occur due to genetic mutations or exposure to certain environmental factors. Some risk factors that have been linked to brain stem neoplasms include:

* Family history of cancer
* Exposure to radiation therapy in childhood
* Previous head trauma
* Certain genetic conditions, such as turcot syndrome

Symptoms:

The symptoms of brain stem neoplasms can vary depending on their size, location, and severity. Some common symptoms include:

* Headaches
* Vision problems
* Weakness or numbness in the limbs
* Slurred speech
* Difficulty with balance and coordination
* Seizures
* Hydrocephalus (fluid buildup in the brain)

Diagnosis:

To diagnose a brain stem neoplasm, a doctor will typically perform a physical exam and ask questions about the patient's medical history. They may also order several tests, such as:

* CT or MRI scans to visualize the tumor
* Electroencephalogram (EEG) to measure electrical activity in the brain
* Blood tests to check for certain substances that are produced by the tumor

Treatment options:

The treatment of brain stem neoplasms depends on several factors, including the size and location of the tumor, the patient's age and overall health, and the type of tumor. Some possible treatment options include:

* Surgery to remove the tumor
* Radiation therapy to kill cancer cells
* Chemotherapy to kill cancer cells
* Observation and monitoring for small, slow-growing tumors that do not cause significant symptoms

Prognosis:

The prognosis for brain stem neoplasms varies depending on the type of tumor and the patient's overall health. In general, the prognosis is poor for patients with brain stem tumors, as they can be difficult to treat and may recur. However, with prompt and appropriate treatment, some patients may experience a good outcome.

Lifestyle changes:

There are no specific lifestyle changes that can cure a brain stem neoplasm, but some changes may help improve the patient's quality of life. These may include:

* Avoiding activities that exacerbate symptoms, such as heavy lifting or bending
* Taking regular breaks to rest and relax
* Eating a healthy diet and getting plenty of sleep
* Reducing stress through techniques such as meditation or deep breathing exercises.

It's important for patients with brain stem neoplasms to work closely with their healthcare team to manage their symptoms and monitor their condition. With prompt and appropriate treatment, some patients may experience a good outcome.

Examples of infratentorial neoplasms include:

1. Cerebellar astrocytomas
2. Brain stem gliomas
3. Vestibular schwannomas (acoustic neuromas)
4. Meningiomas
5. Metastatic tumors to the infratentorial region

Infratentorial neoplasms can cause a wide range of symptoms depending on their size, location, and degree of malignancy. Common symptoms include headache, nausea, vomiting, balance problems, weakness or numbness in the arms or legs, double vision, and difficulty with speech or swallowing.

Infratentorial neoplasms are diagnosed using a combination of imaging techniques such as CT or MRI scans, and tissue biopsy may be necessary to confirm the diagnosis. Treatment options for infratentorial neoplasms depend on the type, size, and location of the tumor, but may include surgery, radiation therapy, and chemotherapy.

The term "papilledema" comes from the Greek words "papilla," meaning "little nipple," and "dema," meaning "swelling." This refers to the appearance of the optic disc when it is swollen, as it looks like a small, round nipple on the surface of the retina.

Papilledema can be caused by a variety of conditions, including high blood pressure, brain tumors, and aneurysms. It can also be a symptom of other conditions such as meningitis or multiple sclerosis. The diagnosis of papilledema is typically made through a comprehensive eye exam, which includes visual acuity testing, refraction, and retinoscopy. Imaging tests such as MRI or CT scans may also be used to evaluate the cause of the swelling.

Treatment of papilledema depends on the underlying cause of the condition. In cases where high blood pressure is the cause, medication to lower blood pressure may be prescribed. In other cases, surgery or other interventions may be necessary to relieve pressure on the brain and reduce swelling in the optic disc.

It's important for individuals with papilledema to work closely with their healthcare provider to monitor and manage their condition, as untreated papilledema can lead to permanent vision loss.

The symptoms of GBS can range from mild to severe and may include:

* Weakness or tingling sensations in the legs, arms, or face
* Muscle weakness that progresses to paralysis
* Loss of reflexes
* Difficulty swallowing or speaking
* Numbness or pain in the hands and feet
* Fatigue and fever

The diagnosis of GBS is based on a combination of symptoms, physical examination findings, and laboratory tests. There is no cure for GBS, but treatment can help manage symptoms and prevent complications. Plasmapheresis, immunoglobulin therapy, and corticosteroids are common treatments used to reduce inflammation and slow the progression of the disease.

GBS is a rare condition that affects about one in 100,000 people per year in the United States. It can affect anyone, but it is more common in children and young adults. The prognosis for GBS varies depending on the severity of the disease, but most people recover fully within a few weeks or months with proper treatment.

In conclusion, Guillain-Barré Syndrome is a rare autoimmune disorder that can cause muscle weakness and paralysis. While there is no cure for GBS, early diagnosis and treatment can help manage symptoms and prevent complications. With proper care, most people with GBS can recover fully within a few weeks or months.

There are several types of diabetic neuropathies, including:

1. Peripheral neuropathy: This is the most common type of diabetic neuropathy and affects the nerves in the hands and feet. It can cause numbness, tingling, and pain in these areas.
2. Autonomic neuropathy: This type of neuropathy affects the nerves that control involuntary functions, such as digestion, bladder function, and blood pressure. It can cause a range of symptoms, including constipation, diarrhea, urinary incontinence, and sexual dysfunction.
3. Proximal neuropathy: This type of neuropathy affects the nerves in the legs and hips. It can cause weakness, pain, and stiffness in these areas.
4. Focal neuropathy: This type of neuropathy affects a single nerve, often causing sudden and severe pain.

The exact cause of diabetic neuropathies is not fully understood, but it is thought to be related to high blood sugar levels over time. Other risk factors include poor blood sugar control, obesity, smoking, and alcohol consumption. There is no cure for diabetic neuropathy, but there are several treatments available to manage the symptoms and prevent further nerve damage. These treatments may include medications, physical therapy, and lifestyle changes such as regular exercise and a healthy diet.

Some common neurological manifestations include:

1. Weakness or paralysis of specific muscle groups
2. Numbness or tingling sensations in the limbs or body
3. Difficulty with speech, language, or swallowing
4. Vision problems, such as blurred vision, double vision, or loss of vision
5. Dizziness, vertigo, or loss of balance
6. Confusion, disorientation, or difficulty with memory
7. Seizures or convulsions
8. Headaches or migraines
9. Sleep disturbances, such as insomnia or narcolepsy
10. Behavioral changes, such as mood swings, depression, or anxiety

The neurological manifestations of a condition can vary depending on the underlying cause and the specific location and extent of the damage to the nervous system. Diagnosis and treatment of these manifestations require expertise in neurology and may involve a range of diagnostic tests, such as imaging studies, electromyography, and laboratory tests, as well as medications, surgery, or other interventions.

Examples of abnormal reflexes include:

1. Overactive reflexes: Reflexes that are too strong or exaggerated, such as an oversensitive knee jerk reflex.
2. Underactive reflexes: Reflexes that are too weak or diminished, such as a decreased tendon reflex in the arm.
3. Delayed reflexes: Reflexes that take longer than expected to occur, such as a delayed deep tendon reflex.
4. Abnormal reflex arc: A reflex arc that is not normal or expected for the situation, such as a spastic reflex arc.
5. Reflexes that are out of proportion to the stimulus: Such as an excessive or exaggerated reflex response to a mild stimulus.
6. Reflexes that occur in the absence of a stimulus: Such as a spontaneous reflex.
7. Reflexes that do not resolve: Such as a persistent reflex.
8. Reflexes that are painful or uncomfortable: Such as an abnormal rectal reflex.

It's important to note that not all abnormal reflexes are necessarily indicative of a serious medical condition, but they should be evaluated by a healthcare professional to determine the underlying cause and appropriate treatment.

Bulbar palsy, progressive refers to a condition where there is a gradual loss of muscle function in the face, tongue, and throat due to damage to the brainstem. This condition is also known as progressive bulbar palsy (PBP).

The brainstem is responsible for controlling many of the body's automatic functions, including breathing, heart rate, and swallowing. When the brainstem is damaged, it can lead to a range of symptoms, including weakness or paralysis of the muscles in the face, tongue, and throat.

The symptoms of progressive bulbar palsy may include:

* Difficulty speaking or slurred speech
* Weakness or paralysis of the facial muscles
* Difficulty swallowing (dysphagia)
* Weight loss due to difficulty eating and drinking
* Fatigue and weakness
* Decreased reflexes

Progressive bulbar palsy can be caused by a variety of conditions, including:

* Brainstem stroke or bleeding
* Brain tumors
* Multiple sclerosis
* Amyotrophic lateral sclerosis (ALS)
* Other neurodegenerative disorders

There is no cure for progressive bulbar palsy, but treatment may include:

* Speech therapy to improve communication skills
* Swallowing therapy to reduce the risk of choking or pneumonia
* Physical therapy to maintain muscle strength and function
* Medications to manage symptoms such as pain, weakness, or fatigue

The prognosis for progressive bulbar palsy is generally poor, with many individuals experiencing significant decline in their quality of life and eventually succumbing to the disease. However, the rate of progression can vary greatly depending on the underlying cause of the condition.

Intracranial aneurysms are relatively rare but can have serious consequences if they rupture and cause bleeding in the brain.

The symptoms of an unruptured intracranial aneurysm may include headaches, seizures, and visual disturbances.

If an intracranial aneurysm ruptures, it can lead to a subarachnoid hemorrhage (bleeding in the space around the brain), which is a medical emergency that requires immediate treatment.

Diagnosis of an intracranial aneurysm typically involves imaging tests such as CT or MRI scans, and may also involve catheter angiography.

Treatment for intracranial aneurysms usually involves surgical clipping or endovascular coiling, depending on the size, location, and severity of the aneurysm.

Preventing rupture of intracranial aneurysms is important, as they can be difficult to treat once they have ruptured.

Endovascular coiling is a minimally invasive procedure in which a catheter is inserted into the affected artery and a small coil is inserted into the aneurysm, causing it to clot and preventing further bleeding.

Surgical clipping involves placing a small metal clip across the base of the aneurysm to prevent further bleeding.

In addition to these treatments, medications such as anticonvulsants and antihypertensives may be used to manage symptoms and prevent complications.

Treatment involves administration of anti-TB drugs, usually in combination with supportive care to manage symptoms and prevent complications such as seizures and brain damage. Treatment can take several months and must be completed even if symptoms improve before finishing treatment.

Prevention is difficult because TB bacteria are often resistant to standard antibiotics, so it's important for individuals with HIV or other conditions that weaken the immune system to avoid exposure to TB bacteria whenever possible and receive regular screening tests.

There are many different types of nerve degeneration that can occur in various parts of the body, including:

1. Alzheimer's disease: A progressive neurological disorder that affects memory and cognitive function, leading to degeneration of brain cells.
2. Parkinson's disease: A neurodegenerative disorder that affects movement and balance, caused by the loss of dopamine-producing neurons in the brain.
3. Amyotrophic lateral sclerosis (ALS): A progressive neurological disease that affects nerve cells in the brain and spinal cord, leading to muscle weakness, paralysis, and eventually death.
4. Multiple sclerosis: An autoimmune disease that affects the central nervous system, causing inflammation and damage to nerve fibers.
5. Diabetic neuropathy: A complication of diabetes that can cause damage to nerves in the hands and feet, leading to pain, numbness, and weakness.
6. Guillain-Barré syndrome: An autoimmune disorder that can cause inflammation and damage to nerve fibers, leading to muscle weakness and paralysis.
7. Chronic inflammatory demyelinating polyneuropathy (CIDP): An autoimmune disorder that can cause inflammation and damage to nerve fibers, leading to muscle weakness and numbness.

The causes of nerve degeneration are not always known or fully understood, but some possible causes include:

1. Genetics: Some types of nerve degeneration may be inherited from one's parents.
2. Aging: As we age, our nerve cells can become damaged or degenerate, leading to a decline in cognitive and physical function.
3. Injury or trauma: Physical injury or trauma to the nervous system can cause nerve damage and degeneration.
4. Infections: Certain infections, such as viral or bacterial infections, can cause nerve damage and degeneration.
5. Autoimmune disorders: Conditions such as Guillain-Barré syndrome and chronic inflammatory demyelinating polyneuropathy (CIDP) are caused by the immune system attacking and damaging nerve cells.
6. Toxins: Exposure to certain toxins, such as heavy metals or pesticides, can damage and degenerate nerve cells.
7. Poor nutrition: A diet that is deficient in essential nutrients, such as vitamin B12 or other B vitamins, can lead to nerve damage and degeneration.
8. Alcoholism: Long-term alcohol abuse can cause nerve damage and degeneration due to the toxic effects of alcohol on nerve cells.
9. Drug use: Certain drugs, such as chemotherapy drugs and antiviral medications, can damage and degenerate nerve cells.
10. Aging: As we age, our nerve cells can deteriorate and become less functional, leading to a range of cognitive and motor symptoms.

It's important to note that in some cases, nerve damage and degeneration may be irreversible, but there are often strategies that can help manage symptoms and improve quality of life. If you suspect you have nerve damage or degeneration, it's important to seek medical attention as soon as possible to receive an accurate diagnosis and appropriate treatment.

Platybasia can be caused by a variety of factors, including:

1. Chronic inflammation: Prolonged inflammation can cause the basal cells to flatten and spread out, leading to platybasia.
2. Infection: Certain infections, such as herpes simplex virus, can cause platybasia by damaging the epithelial cells.
3. Irritation: Repeated irritation or trauma to the skin or mucous membranes can lead to platybasia.
4. Genetic disorders: Certain genetic disorders, such as epidermolysis bullosa, can cause platybasia by impairing the ability of the epithelial cells to adhere to each other.
5. Cancer: Platybasia can be a feature of some types of cancer, such as squamous cell carcinoma.

The symptoms of platybasia can vary depending on the location and severity of the condition. They may include:

1. Redness and inflammation
2. Thickening of the skin or mucous membranes
3. Formation of scaly or crusted lesions
4. Discharge or bleeding from the affected area
5. Pain or discomfort

The diagnosis of platybasia is typically made through a combination of physical examination, medical history, and diagnostic tests such as biopsy or imaging studies. Treatment depends on the underlying cause of the condition and may include antibiotics, topical medications, or surgery.

In summary, platybasia is a condition characterized by the flattening and spreading out of basal cells in the epithelium, which can be caused by a variety of factors and can occur in various parts of the body. It can cause a range of symptoms and may be associated with certain medical conditions or cancer. Accurate diagnosis and appropriate treatment are important to prevent complications and improve outcomes.

Causes:

The exact cause of SST is not well understood, but it is believed to be related to abnormal blood flow or coagulation disorders. Some possible causes include:

* Infection: Bacterial, viral, or fungal infections can cause inflammation and damage to the blood vessels in the brain, leading to the formation of a clot in the sagittal sinus.
* Trauma: Head injuries or other types of trauma can cause damage to the blood vessels in the brain, leading to the formation of a clot in the sagittal sinus.
* Genetic predisposition: Some people may be born with a genetic predisposition to develop SST.
* Cancer: Certain types of cancer, such as lymphoma or leukemia, can cause abnormal blood flow and increase the risk of SST.

Symptoms:

The symptoms of SST can vary depending on the location and size of the clot. Some common symptoms include:

* Headache: A severe headache is often the first symptom of SST, which may be accompanied by nausea and vomiting.
* Seizures: SST can cause seizures, especially in children.
* Confusion: Patients with SST may experience confusion, disorientation, and difficulty concentrating.
* Weakness or paralysis: Depending on the location of the clot, patients may experience weakness or paralysis in the arms, legs, or face.
* Vision problems: SST can cause vision problems, including blurred vision, double vision, or loss of peripheral vision.

Diagnosis:

SST is difficult to diagnose, as the symptoms can be similar to other conditions such as meningitis or stroke. A thorough physical examination and imaging studies are necessary to confirm the diagnosis. Some common diagnostic tests include:

* Computed tomography (CT) scan: A CT scan can help identify the location and size of the clot.
* Magnetic resonance imaging (MRI): An MRI can provide more detailed information about the clot and its effects on surrounding tissue.
* Doppler ultrasound: A Doppler ultrasound can help identify blood flow in the affected area.

Treatment:

The treatment of SST depends on the location and size of the clot, as well as the underlying cause. Some common treatment options include:

* Anticoagulant medications: These medications, such as heparin or warfarin, can help prevent further clotting.
* Thrombolytic medications: These medications, such as tissue plasminogen activator (tPA), can dissolve the clot.
* Surgery: In some cases, surgery may be necessary to remove the clot or repair damaged blood vessels.

Complications:

SST can have serious complications, including:

* Stroke: If the clot blocks the flow of blood to the brain, it can cause a stroke.
* Heart attack: If the clot blocks the flow of blood to the heart, it can cause a heart attack.
* Pulmonary embolism: If the clot breaks loose and travels to the lungs, it can cause a pulmonary embolism.

Prevention:

To prevent SST, it is important to take steps to reduce your risk factors. This can include:

* Controlling high blood pressure
* Controlling high cholesterol levels
* Quitting smoking
* Maintaining a healthy weight
* Exercising regularly
* Avoiding long periods of immobility, such as during long-distance travel.

Prognosis:

The prognosis for SST is generally good if the clot is detected and treated early. However, if the clot is left untreated, it can lead to serious complications and can be fatal.

Lifestyle changes:

To reduce the risk of developing SST, you can make lifestyle changes such as:

* Quitting smoking
* Maintaining a healthy weight
* Exercising regularly
* Avoiding long periods of immobility, such as during long-distance travel.

Medications:

There are several medications that can be used to treat SST, including:

* Anticoagulants, such as warfarin or heparin, which prevent the clot from growing larger and prevent new clots from forming.
* Thrombolytics, such as tissue plasminogen activator (tPA), which dissolve the clot.
* Antiplatelet agents, such as aspirin, which prevent platelets from sticking together and forming a clot.

Surgery:

In some cases, surgery may be necessary to treat SST. This can include:

* Endovascular therapy, such as angioplasty or stenting, which can be used to open up the blocked blood vessel.
* Bypass surgery, which can be used to reroute blood flow around the blocked blood vessel.

Complications:

SST can cause a number of complications, including:

* Pulmonary embolism: If a piece of the clot breaks loose and travels to the lungs, it can cause a pulmonary embolism, which can be life-threatening.
* Stroke or brain damage: If the clot blocks blood flow to the brain, it can cause a stroke or brain damage.
* Infection: The clot can become infected, which can lead to sepsis and other complications.
* Amputation: In severe cases, SST can lead to tissue death and amputation of the affected limb.

Prognosis:

The prognosis for SST is generally good if the condition is diagnosed and treated promptly. However, if left untreated, it can lead to serious complications and even death. The overall survival rate for SST is about 80%.

Lifestyle Changes:

There are several lifestyle changes that can help reduce the risk of developing SST, including:

* Maintaining a healthy weight
* Exercising regularly
* Avoiding long periods of immobility, such as during long-distance travel.

Medications:

There are several medications that can be used to treat SST, including:

* Anticoagulants, which prevent the clot from growing and prevent new clots from forming.
* Thrombolytics, which dissolve the clot.
* Antiplatelet agents, which prevent platelets from sticking together and forming a clot.

Surgery:

In some cases, surgery may be necessary to treat SST. This can include:

* Thrombectomy, which involves removing the clot.
* Embolectomy, which involves removing the clot from the blood vessel.

Conclusion:

SST is a serious condition that can lead to severe complications if left untreated. However, with prompt diagnosis and treatment, the prognosis is generally good. Lifestyle changes and medications can help reduce the risk of developing SST, and surgery may be necessary in some cases. It is important to seek medical attention immediately if you experience any symptoms of SST.

Symptoms of otitis externa may include:

* Ear pain or tenderness
* Redness and swelling of the ear canal
* Discharge or pus in the ear canal
* Itching or burning sensation in the ear canal
* Fever or chills
* Difficulty hearing or feeling as if the ear is clogged

Otitis externa can be diagnosed by a healthcare professional through a physical examination of the ear canal and may also involve a pus sample or imaging tests such as X-rays or CT scans to rule out other conditions. Treatment options for otitis externa may include antibiotics, anti-inflammatory medications, or topical creams or drops to reduce pain and inflammation. In severe cases, surgery may be necessary to remove any infected tissue or debris from the ear canal.

Prevention of otitis externa includes avoiding exposure to moisture, using earplugs when swimming or showering, and keeping the ears clean and dry. If you suspect you have otitis externa, it is important to seek medical attention promptly to prevent complications such as mastoiditis or meningitis.

There is no cure for tinnitus, but there are several treatment options available to help manage the condition. These include sound therapy, which involves exposing the ear to soothing sounds to mask the tinnitus, and counseling, which can help individuals cope with the emotional effects of tinnitus. Other treatments may include medications to relieve anxiety or depression, relaxation techniques, and lifestyle changes such as avoiding loud noises and taking steps to reduce stress.

It is important for individuals who experience tinnitus to seek medical attention if the condition persists or worsens over time, as it can be a symptom of an underlying medical condition that requires treatment. A healthcare professional can evaluate the individual's hearing and overall health to determine the cause of the tinnitus and develop an appropriate treatment plan.

1. Foodborne botulism: This type of botulism is caused by eating foods that have been contaminated with the bacteria. Symptoms typically begin within 12 to 72 hours after consuming the contaminated food and can include double vision, droopy eyelids, slurred speech, difficulty swallowing, and muscle weakness.
2. Infant botulism: This type of botulism occurs in infants who are exposed to the bacteria through contact with contaminated soil or object. Symptoms can include constipation, poor feeding, and weak cry.
3. Wound botulism: This type of botulism is caused by the bacteria entering an open wound, usually a deep puncture wound or surgical incision.

Botulism is a rare illness in the United States, but it can be deadly if not treated promptly. Treatment typically involves supportive care, such as mechanical ventilation and fluids, as well as antitoxin injections to neutralize the effects of the toxin. Prevention measures include proper food handling and storage, good hygiene practices, and avoiding consumption of improperly canned or preserved foods.

Some common types of cerebellar diseases include:

1. Cerebellar atrophy: This is a condition where the cerebellum shrinks or degenerates, leading to symptoms such as tremors, muscle weakness, and difficulty with movement.
2. Cerebellar degeneration: This is a condition where the cerebellum deteriorates over time, leading to symptoms such as loss of coordination, balance problems, and difficulties with speech and language.
3. Cerebellar tumors: These are abnormal growths that develop in the cerebellum, which can cause a variety of symptoms depending on their size and location.
4. Cerebellar stroke: This is a condition where blood flow to the cerebellum is interrupted, leading to damage to the brain tissue and symptoms such as weakness or paralysis of certain muscle groups.
5. Cerebellar vasculature disorders: These are conditions that affect the blood vessels in the cerebellum, leading to symptoms such as transient ischemic attacks (TIAs) or strokes.
6. Inflammatory diseases: These are conditions that cause inflammation in the cerebellum, leading to symptoms such as tremors, ataxia, and weakness.
7. Infections: Bacterial, viral, or fungal infections can affect the cerebellum and cause a range of symptoms.
8. Trauma: Head injuries or other forms of trauma can damage the cerebellum and lead to symptoms such as loss of coordination, balance problems, and memory loss.
9. Genetic disorders: Certain genetic mutations can affect the development and function of the cerebellum, leading to a range of symptoms.
10. Degenerative diseases: Conditions such as multiple sclerosis, Parkinson's disease, and Huntington's disease can cause degeneration of the cerebellum and lead to symptoms such as tremors, ataxia, and weakness.

It's important to note that this is not an exhaustive list, and there may be other causes of cerebellar symptoms not included here. A healthcare professional can help determine the underlying cause of your symptoms based on a thorough medical history and examination.

The syndrome is named after the doctors who first described it in the 1950s, Drs. Miller and Fisher. It is characterized by a gradual onset of muscle weakness and wasting, which typically begins in the hands and feet and spreads to other parts of the body over time.

Other symptoms of Miller Fisher Syndrome may include:

* Muscle cramps
* Muscle spasms
* Twitching of the eyelids (blepharospasm)
* Loss of reflexes
* Difficulty with speech and swallowing
* Weakness in the muscles of the face, arms, and legs
* Atrophy of the muscles in the hands and feet

The exact cause of Miller Fisher Syndrome is not known, but it is believed to be related to a problem with the nerve cells that control voluntary muscle movement. The disorder usually affects adults between the ages of 50 and 70, and men are more commonly affected than women.

There is no cure for Miller Fisher Syndrome, but treatment can help manage the symptoms. Physical therapy, occupational therapy, and medications such as anticonvulsants and muscle relaxants may be used to improve muscle strength and function. In severe cases, a ventilator may be needed to assist with breathing.

The progression of Miller Fisher Syndrome can vary widely, and some people may experience a rapid decline in muscle function while others may remain relatively stable for many years. The life expectancy of individuals with the disorder is generally reduced due to the risk of complications such as respiratory failure and pneumonia.

Dysarthria can affect both children and adults, and the symptoms can vary in severity depending on the underlying cause of the condition. Some common symptoms of dysarthria include:

* Slurred or slow speech
* Difficulty articulating words
* Poor enunciation
* Stuttering or hesitation while speaking
* Difficulty with word-finding and language processing
* Limited range of speech sounds
* Difficulty with loudness and volume control

Dysarthria can be diagnosed by a speech-language pathologist (SLP), who will typically conduct a comprehensive evaluation of the individual's speech and language abilities. This may include a series of tests to assess the individual's articulation, fluency, voice quality, and other aspects of their speech.

There are several types of dysarthria, including:

* Hypokinetic dysarthria: characterized by reduced muscle tone and slow movement of the articulatory organs, resulting in slurred or slow speech.
* Hyperkinetic dysarthria: characterized by increased muscle tone and rapid movement of the articulatory organs, resulting in fast but imprecise speech.
* Mixed dysarthria: a combination of hypokinetic and hyperkinetic features.
* Dystonic dysarthria: characterized by involuntary movements and postures of the tongue and lips, resulting in distorted speech.

Treatment for dysarthria typically involves speech therapy with an SLP, who will work with the individual to improve their speech clarity, fluency, and overall communication skills. Treatment may include exercises to strengthen the muscles used in speech production, as well as strategies to improve articulation, pronunciation, and language processing. In some cases, technology such as speech-generating devices may be used to support communication.

In addition to speech therapy, treatment for dysarthria may also involve other healthcare professionals, such as neurologists, physical therapists, or occupational therapists, depending on the underlying cause of the condition.

Overall, dysarthria is a speech disorder that can significantly impact an individual's ability to communicate effectively. However, with the right treatment and support from healthcare professionals and SLPs, many people with dysarthria are able to improve their communication skills and lead fulfilling lives.

Nerve sheath neoplasms are usually slow-growing and may not cause any symptoms in the early stages. However, as they grow, they can exert pressure on the surrounding nerve tissue and cause a variety of symptoms, including:

1. Pain or numbness in the affected area
2. Weakness or paralysis of the muscles served by the affected nerve
3. Tingling or burning sensations in the skin or extremities
4. Seizures, in rare cases

The exact cause of nerve sheath neoplasms is not known, but they are thought to be associated with genetic mutations that affect the development and growth of nerve cells. Some cases may also be caused by inherited conditions, such as Neurofibromatosis type 1 (NF1) or schwannomatosis.

There are several types of nerve sheath neoplasms, including:

1. Neurofibromas: These are the most common type of nerve sheath tumor and are usually benign. They can occur in any part of the body and may grow slowly over time.
2. Schwannomas: These are also benign tumors that arise from the covering of nerves (the schwann cells). They are usually slow-growing and can occur in any part of the body.
3. Malignant peripheral nerve sheath tumors (MPNSTs): These are rare and aggressive tumors that can arise from the coverings of nerves. They can grow rapidly and can be difficult to treat.

Diagnosis of nerve sheath neoplasms typically involves a combination of imaging studies, such as MRI or CT scans, and a biopsy to confirm the presence of a tumor. Treatment options vary depending on the type, size, and location of the tumor, as well as the patient's overall health. Surgery is often the first line of treatment for nerve sheath neoplasms, and may be followed by radiation therapy or chemotherapy in some cases.

Peripheral Nervous System Diseases can result from a variety of causes, including:

1. Trauma or injury
2. Infections such as Lyme disease or HIV
3. Autoimmune disorders such as Guillain-Barré syndrome
4. Genetic mutations
5. Tumors or cysts
6. Toxins or poisoning
7. Vitamin deficiencies
8. Chronic diseases such as diabetes or alcoholism

Some common Peripheral Nervous System Diseases include:

1. Neuropathy - damage to the nerves that can cause pain, numbness, and weakness in the affected areas.
2. Multiple Sclerosis (MS) - an autoimmune disease that affects the CNS and PNS, causing a range of symptoms including numbness, weakness, and vision problems.
3. Peripheral Neuropathy - damage to the nerves that can cause pain, numbness, and weakness in the affected areas.
4. Guillain-Barré syndrome - an autoimmune disorder that causes muscle weakness and paralysis.
5. Charcot-Marie-Tooth disease - a group of inherited disorders that affect the nerves in the feet and legs, leading to muscle weakness and wasting.
6. Friedreich's ataxia - an inherited disorder that affects the nerves in the spine and limbs, leading to coordination problems and muscle weakness.
7. Chronic Inflammatory Demyelinating Polyneuropathy (CIDP) - an autoimmune disorder that causes inflammation of the nerves, leading to pain, numbness, and weakness in the affected areas.
8. Amyotrophic Lateral Sclerosis (ALS) - a progressive neurological disease that affects the nerve cells responsible for controlling voluntary muscle movement, leading to muscle weakness, atrophy, and paralysis.
9. Spinal Muscular Atrophy - an inherited disorder that affects the nerve cells responsible for controlling voluntary muscle movement, leading to muscle weakness and wasting.
10. Muscular Dystrophy - a group of inherited disorders that affect the nerve cells responsible for controlling voluntary muscle movement, leading to muscle weakness and wasting.

It's important to note that this is not an exhaustive list and there may be other causes of muscle weakness. If you are experiencing persistent or severe muscle weakness, it is important to see a healthcare professional for proper evaluation and diagnosis.

The term "Bell's palsy" was coined by Sir Charles Bell in 1829, and it is named after him. The condition is caused by damage to the facial nerve, which can be due to a variety of factors such as viral infections, autoimmune disorders, trauma, or tumors.

Bell's palsy can cause a range of symptoms including:

* Weakness or paralysis of the facial muscles on one side of the face
* Drooping or weakness of the eyelid or corner of the mouth
* Difficulty closing the eye or smiling
* Dryness or excessive tearing of the eye
* Increased sensitivity to sound or touch on the affected side
* Pain or discomfort in the face, jaw, or ear

Bell's palsy can be diagnosed by a neurologist based on symptoms and physical examination. Imaging tests such as MRI or CT scans may be ordered to rule out other conditions that can cause similar symptoms.

There is no cure for Bell's palsy, but various treatments can help manage the symptoms. These may include:

* Medications to reduce inflammation and pain
* Physical therapy to improve facial function and appearance
* Surgery to repair or remove damaged nerve tissue
* Corticosteroid injections to reduce swelling and inflammation

The prognosis for Bell's palsy is generally good, with most people experiencing a full recovery within a few weeks to months. However, some people may experience long-term symptoms or complications such as permanent nerve damage or eye dryness.

Some common types of brain diseases include:

1. Neurodegenerative diseases: These are progressive conditions that damage or kill brain cells over time, leading to memory loss, cognitive decline, and movement disorders. Examples include Alzheimer's disease, Parkinson's disease, Huntington's disease, and amyotrophic lateral sclerosis (ALS).
2. Stroke: This occurs when blood flow to the brain is interrupted, leading to cell death and potential long-term disability.
3. Traumatic brain injury (TBI): This refers to any type of head injury that causes damage to the brain, such as concussions, contusions, or penetrating wounds.
4. Infections: Viral, bacterial, and fungal infections can all affect the brain, leading to a range of symptoms including fever, seizures, and meningitis.
5. Tumors: Brain tumors can be benign or malignant and can cause a variety of symptoms depending on their location and size.
6. Cerebrovascular diseases: These conditions affect the blood vessels of the brain, leading to conditions such as aneurysms, arteriovenous malformations (AVMs), and Moyamoya disease.
7. Neurodevelopmental disorders: These are conditions that affect the development of the brain and nervous system, such as autism spectrum disorder, ADHD, and intellectual disability.
8. Sleep disorders: Conditions such as insomnia, narcolepsy, and sleep apnea can all have a significant impact on brain function.
9. Psychiatric disorders: Mental health conditions such as depression, anxiety, and schizophrenia can affect the brain and its functioning.
10. Neurodegenerative with brain iron accumulation: Conditions such as Parkinson's disease, Alzheimer's disease, and Huntington's disease are characterized by the accumulation of abnormal proteins and other substances in the brain, leading to progressive loss of brain function over time.

It is important to note that this is not an exhaustive list and there may be other conditions or factors that can affect the brain and its functioning. Additionally, many of these conditions can have a significant impact on a person's quality of life, and it is important to seek medical attention if symptoms persist or worsen over time.

Symptoms of meningitis may include fever, headache, stiff neck, confusion, nausea and vomiting, and sensitivity to light. In severe cases, it can lead to seizures, brain damage, and even death.

There are several types of meningitis, including:

1. Viral meningitis: This is the most common form of the infection and is usually caused by enteroviruses or herpesviruses. It is typically less severe than bacterial meningitis and resolves on its own with supportive care.
2. Bacterial meningitis: This is a more serious form of the infection and can be caused by a variety of bacteria, such as Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae. It requires prompt antibiotic treatment to prevent long-term complications and death.
3. Fungal meningitis: This type of meningitis is more common in people with weakened immune systems and is caused by fungi that are commonly found in the environment. It can be treated with antifungal medications.
4. Parasitic meningitis: This type of meningitis is rare and is caused by parasites that are typically found in tropical regions. It can be treated with antiparasitic medications.

Diagnosis of meningitis is based on a combination of clinical findings, laboratory tests, and imaging studies. Laboratory tests may include blood cultures, polymerase chain reaction (PCR) testing, and cerebrospinal fluid (CSF) analysis. Imaging studies, such as CT or MRI scans, may be used to rule out other conditions and to evaluate the extent of brain damage.

Treatment of meningitis depends on the cause of the infection and may include antibiotics, antiviral medications, antifungal medications, or supportive care to manage symptoms and prevent complications. Supportive care may include intravenous fluids, oxygen therapy, and pain management. In severe cases, meningitis may require hospitalization in an intensive care unit (ICU) and may result in long-term consequences such as hearing loss, learning disabilities, or cognitive impairment.

Prevention of meningitis includes vaccination against the bacteria or viruses that can cause the infection, good hygiene practices, and avoiding close contact with people who are sick. Vaccines are available for certain types of meningitis, such as the meningococcal conjugate vaccine (MenACWY) and the pneumococcal conjugate vaccine (PCV). Good hygiene practices include washing hands frequently, covering the mouth and nose when coughing or sneezing, and avoiding sharing food, drinks, or personal items.

In conclusion, meningitis is a serious and potentially life-threatening infection that can affect people of all ages. Early diagnosis and treatment are crucial to prevent long-term consequences and improve outcomes. Prevention includes vaccination, good hygiene practices, and avoiding close contact with people who are sick.



Signs and Symptoms:

The signs and symptoms of BSI vary depending on the severity and location of the infarction. Common symptoms include sudden onset of headache, confusion, dizziness, slurred speech, weakness or paralysis of the face or limbs, double vision, and difficulty with swallowing. Patients may also experience vomiting, seizures, and loss of consciousness.

Diagnosis:

BSI is diagnosed using a combination of physical examination, imaging studies such as CT or MRI scans, and laboratory tests. A complete neurological examination is crucial to identify any deficits in vision, hearing, balance, and sensation. Imaging studies are used to confirm the presence of an infarction and to identify the location and extent of the damage. Laboratory tests such as blood chemistry and coagulation studies may be performed to rule out other conditions that can cause similar symptoms.

Treatment:

The treatment of BSI depends on the underlying cause and the severity of the infarction. In some cases, surgery may be necessary to relieve the blockage or to repair any blood vessel damage. Medications such as anticoagulants, antiplatelet agents, and blood pressure-lowering drugs may also be used to manage the condition. Rehabilitation therapy is often necessary to help patients regain lost function and improve their quality of life.

Prognosis:

The prognosis for BSI varies depending on the severity and location of the infarction, as well as the underlying cause. In general, patients with a small infarct in a critical area of the brainstem have a poorer prognosis than those with larger infarctions in less critical areas. However, early recognition and treatment can improve outcomes and reduce the risk of complications such as seizures, hydrocephalus, and respiratory failure.

Complications:

BSI can be associated with a number of complications, including:

1. Seizures: BSI can cause seizures, which can be challenging to treat and may require medication or surgical intervention.
2. Hydrocephalus: Fluid buildup in the brain can occur as a result of BSI, leading to increased intracranial pressure and potentially life-threatening complications.
3. Respiratory failure: Damage to the brainstem can lead to respiratory failure, which may require mechanical ventilation.
4. Cardiac arrhythmias: BSI can cause cardiac arrhythmias, which can be life-threatening if not treated promptly.
5. Cerebral edema: Swelling in the brain can occur as a result of BSI, leading to increased intracranial pressure and potentially life-threatening complications.
6. Pneumonia: BSI can increase the risk of developing pneumonia, particularly in individuals with pre-existing respiratory conditions.
7. Meningitis: BSI can increase the risk of developing meningitis, particularly in individuals with pre-existing immune compromise.
8. Stroke: BSI can cause stroke, which may be related to the infarction itself or to the underlying condition that caused the infarction.
9. Cognitive and behavioral changes: BSI can result in cognitive and behavioral changes, including difficulty with concentration, memory loss, and personality changes.
10. Long-term sequelae: BSI can have long-term consequences, including chronic cognitive impairment, seizures, and changes in behavior and mood.

Treatment and management:

The treatment and management of BSI depend on the underlying cause and the severity of the infarction. Some common approaches include:

1. Antibiotics: If the infarction is caused by an infection, antibiotics may be prescribed to treat the infection and prevent further spread of the infection.
2. Supportive care: Patients with BSI may require supportive care, such as mechanical ventilation, dialysis, or cardiac support, depending on the severity of the infarction.
3. Surgical intervention: In some cases, surgical intervention may be necessary to relieve pressure or remove infected tissue.
4. Rehabilitation: Patients who survive BSI may require rehabilitation to regain lost function and improve their quality of life.
5. Close monitoring: Patients with BSI should be closely monitored for signs of complications, such as seizures, confusion, or changes in vital signs.

Prevention:

Preventing BSI is critical to reducing the risk of complications and improving outcomes. Some strategies for preventing BSI include:

1. Immunization: Vaccination against Streptococcus pneumoniae and Haemophilus influenzae type b can help prevent BSI caused by these organisms.
2. Proper hygiene: Proper hand washing and hygiene practices can help reduce the risk of transmission of BSI-causing pathogens.
3. Use of contact precautions: Use of contact precautions, such as wearing gloves and gowns, can help prevent the spread of BSI-causing pathogens.
4. Proper use of invasive devices: Proper use of invasive devices, such as central lines and urinary catheters, can help reduce the risk of BSI.
5. Antibiotic stewardship: Proper use of antibiotics can help reduce the risk of BSI caused by antibiotic-resistant pathogens.
6. Early detection and treatment: Early detection and treatment of underlying infections can help prevent the progression to BSI.
7. Avoiding unnecessary invasive procedures: Avoiding unnecessary invasive procedures, such as central lines or urinary catheters, can reduce the risk of BSI.
8. Use of antimicrobial-impregnated devices: Use of antimicrobial-impregnated devices, such as central lines and urinary catheters, can help reduce the risk of BSI.
9. Proper hand hygiene: Proper hand hygiene practices, including hand washing and use of alcohol-based hand sanitizers, can help reduce the transmission of BSI-causing pathogens.
10. Environmental cleaning and disinfection: Regular environmental cleaning and disinfection can help reduce the presence of BSI-causing pathogens in the hospital environment.

It is important to note that these strategies should be tailored to the specific needs of each patient and healthcare facility, and may need to be adjusted based on the local prevalence of BSI-causing pathogens and the patient's medical condition.

1. Neurodegenerative diseases: These are diseases that cause progressive loss of brain cells, leading to cognitive decline and motor dysfunction. Examples include Alzheimer's disease, Parkinson's disease, and Huntington's disease.
2. Stroke: A stroke occurs when blood flow to the brain is interrupted, leading to cell death and potential long-term disability.
3. Traumatic brain injury: This type of injury occurs when the brain is subjected to a sudden and forceful impact, such as in a car accident or fall.
4. Infections: Bacterial, viral, and fungal infections can all cause CNS diseases, such as meningitis and encephalitis.
5. Autoimmune disorders: These are conditions in which the immune system mistakenly attacks healthy cells in the brain, leading to inflammation and damage. Examples include multiple sclerosis and lupus.
6. Brain tumors: Tumors can occur in any part of the brain and can be benign or malignant.
7. Cerebrovascular diseases: These are conditions that affect the blood vessels in the brain, such as aneurysms and arteriovenous malformations (AVMs).
8. Neurodevelopmental disorders: These are conditions that affect the development of the brain and nervous system, such as autism spectrum disorder and attention deficit hyperactivity disorder (ADHD).

CNS diseases can have a significant impact on quality of life, and some can be fatal. Treatment options vary depending on the specific diagnosis and severity of the disease. Some CNS diseases can be managed with medication, while others may require surgery or other interventions.

The symptoms of pituitary apoplexy can vary depending on the severity of the condition, but may include:

1. Sudden and severe headache
2. Vision loss or blurriness
3. Nausea and vomiting
4. Confusion and disorientation
5. Weakness or numbness in the face or limbs
6. Seizures
7. Loss of consciousness

The exact cause of pituitary apoplexy is not always clear, but it can be triggered by a variety of factors such as:

1. Trauma to the head or neck
2. Tumors of the pituitary gland or hypothalamus
3. Infection of the pituitary gland (such as meningitis)
4. Hemorrhage (bleeding) within the pituitary gland
5. Radiation therapy or surgery in the brain

Pituitary apoplexy is a medical emergency and requires immediate attention. Treatment typically involves supportive care, such as fluid replacement and pain management, as well as addressing any underlying causes. In some cases, surgery may be necessary to relieve compression on the pituitary gland or to remove a tumor.

Early diagnosis and treatment are critical for improving outcomes in patients with pituitary apoplexy. However, the condition can be challenging to diagnose due to its rarity and non-specific symptoms. A thorough clinical evaluation, including neurological examination and imaging studies such as MRI or CT scans, is necessary for accurate diagnosis.

There are many different types of eye diseases, including:

1. Cataracts: A clouding of the lens in the eye that can cause blurry vision and blindness.
2. Glaucoma: A group of diseases that damage the optic nerve and can lead to vision loss and blindness.
3. Age-related macular degeneration (AMD): A condition that causes vision loss in older adults due to damage to the macula, the part of the retina responsible for central vision.
4. Diabetic retinopathy: A complication of diabetes that can cause damage to the blood vessels in the retina and lead to vision loss.
5. Detached retina: A condition where the retina becomes separated from the underlying tissue, leading to vision loss.
6. Macular hole: A small hole in the macula that can cause vision loss.
7. Amblyopia (lazy eye): A condition where one eye is weaker than the other and has reduced vision.
8. Strabismus (crossed eyes): A condition where the eyes are not aligned properly and point in different directions.
9. Conjunctivitis: An inflammation of the conjunctiva, the thin membrane that covers the white part of the eye and the inside of the eyelids.
10. Dry eye syndrome: A condition where the eyes do not produce enough tears, leading to dryness, itchiness, and irritation.

Eye diseases can be caused by a variety of factors, including genetics, age, environmental factors, and certain medical conditions. Some eye diseases are inherited, while others are acquired through lifestyle choices or medical conditions.

Symptoms of eye diseases can include blurry vision, double vision, eye pain, sensitivity to light, and redness or inflammation in the eye. Treatment options for eye diseases depend on the specific condition and can range from medication, surgery, or lifestyle changes.

Regular eye exams are important for detecting and managing eye diseases, as many conditions can be treated more effectively if caught early. If you experience any symptoms of eye disease or have concerns about your vision, it is important to see an eye doctor as soon as possible.

Examples of syndromes include:

1. Down syndrome: A genetic disorder caused by an extra copy of chromosome 21 that affects intellectual and physical development.
2. Turner syndrome: A genetic disorder caused by a missing or partially deleted X chromosome that affects physical growth and development in females.
3. Marfan syndrome: A genetic disorder affecting the body's connective tissue, causing tall stature, long limbs, and cardiovascular problems.
4. Alzheimer's disease: A neurodegenerative disorder characterized by memory loss, confusion, and changes in personality and behavior.
5. Parkinson's disease: A neurological disorder characterized by tremors, rigidity, and difficulty with movement.
6. Klinefelter syndrome: A genetic disorder caused by an extra X chromosome in males, leading to infertility and other physical characteristics.
7. Williams syndrome: A rare genetic disorder caused by a deletion of genetic material on chromosome 7, characterized by cardiovascular problems, developmental delays, and a distinctive facial appearance.
8. Fragile X syndrome: The most common form of inherited intellectual disability, caused by an expansion of a specific gene on the X chromosome.
9. Prader-Willi syndrome: A genetic disorder caused by a defect in the hypothalamus, leading to problems with appetite regulation and obesity.
10. Sjogren's syndrome: An autoimmune disorder that affects the glands that produce tears and saliva, causing dry eyes and mouth.

Syndromes can be diagnosed through a combination of physical examination, medical history, laboratory tests, and imaging studies. Treatment for a syndrome depends on the underlying cause and the specific symptoms and signs presented by the patient.

Epidermal cysts are relatively common and can occur anywhere on the body, but they are most commonly found on the face, neck, torso, and arms. They are usually small, ranging in size from a pinpoint to a pea-sized bump, but they can sometimes grow larger.

Epidermal cysts are benign (non-cancerous) growths, and they typically do not cause any symptoms unless they become infected or rupture. In rare cases, epidermal cysts can become inflamed or infected, which can lead to redness, swelling, and pain.

Epidermal cysts are usually diagnosed by a dermatologist or other healthcare provider based on their appearance and location. In some cases, a biopsy may be performed to confirm the diagnosis. Treatment for epidermal cysts is usually not necessary unless they become inflamed or infected, in which case antibiotics or surgical drainage may be recommended.

It's important to note that epidermal cysts are different from sebaceous cysts, which are similar but occur in the deeper layers of the skin and are filled with a thick, cheesy material. Both types of cysts are benign and typically do not cause any symptoms unless they become infected or rupture.

There are several types of strabismus, including:

* Esotropia: where one eye turns inward toward the nose
* Exotropia: where one eye turns outward away from the face
* Hypertropia: where one eye turns upward
* Hypotropia: where one eye turns downward
* Duane's syndrome: a rare type of strabismus that affects only one eye and is caused by nerve damage.

Strabismus can have both visual and social consequences, including:

* Difficulty with depth perception and binocular vision
* Blurred or double vision
* Difficulty with eye teaming and tracking
* Poor eye-hand coordination
* Social and emotional effects such as low self-esteem, anxiety, and depression.

Treatment options for strabismus include:

* Glasses or contact lenses to correct refractive errors
* Prism lenses to align the eyes
* Eye exercises to strengthen the muscles and improve eye teaming
* Surgery to adjust the position of the muscles that control eye movement.

It is important for individuals with strabismus to receive timely and appropriate treatment to address the underlying cause of the condition and prevent long-term vision loss and social difficulties.

The most common carotid artery disease is atherosclerosis, which is the buildup of plaque in the inner lining of the arteries. This buildup can lead to a narrowing or blockage of the arteries, reducing blood flow to the brain and increasing the risk of stroke. Other conditions that can affect the carotid arteries include:

1. Carotid artery stenosis: A narrowing of the carotid arteries caused by atherosclerosis or other factors.
2. Carotid artery dissection: A tear in the inner lining of the arteries that can cause bleeding and blockage.
3. Carotid artery aneurysm: A bulge in the wall of the arteries that can lead to rupture and stroke.
4. Temporal bone fracture: A break in the bones of the skull that can cause damage to the carotid arteries and result in stroke or other complications.

Carotid artery diseases are typically diagnosed using imaging tests such as ultrasound, computed tomography (CT) angiography, or magnetic resonance angiography (MRA). Treatment options for carotid artery diseases depend on the underlying condition and its severity, but may include lifestyle changes, medications, surgery, or endovascular procedures.

Prevention of carotid artery diseases is key to reducing the risk of stroke and other complications. This includes managing risk factors such as high blood pressure, high cholesterol, smoking, and diabetes, as well as maintaining a healthy lifestyle and getting regular check-ups with your doctor.

Sarcoidosis is characterized by an abnormal immune response, which leads to the formation of granulomas. These granulomas are made up of a mix of immune cells, including macrophages, lymphocytes, and epithelioid cells. The exact cause of sarcoidosis is not known, but it is believed that a combination of genetic and environmental factors may contribute to its development.

There are several types of sarcoidosis, each with different symptoms and characteristics:

* Cutaneous sarcoidosis affects the skin and can cause red or purple patches on the face, arms, or legs.
* Lung sarcoidosis is the most common form of the disease and can cause shortness of breath, coughing, and chest pain.
* Ocular sarcoidosis can affect the eyes and cause blurred vision, sensitivity to light, and eye pain.
* Cardiac sarcoidosis can affect the heart and cause arrhythmias, heart failure, or cardiac arrest.

There is no cure for sarcoidosis, but treatment options are available to manage symptoms and prevent complications. Treatment options may include medications such as corticosteroids, immunosuppressive drugs, and biologics, as well as lifestyle changes such as exercise and stress management. In severe cases, surgery or other procedures may be necessary to remove affected tissue or organs.

Overall, sarcoidosis is a complex and debilitating disease that can affect various parts of the body. While there is no cure, with proper treatment and self-care, many people with sarcoidosis are able to manage their symptoms and lead active lives.

There are several different types of skull fractures, including:

1. Linear skull fractures: These are fractures that occur in a straight line and do not involve the brain.
2. Depressed skull fractures: These are fractures that cause the bone to be pushed inward, creating a depression in the skull.
3. Comminuted skull fractures: These are fractures that involve multiple pieces of bone breaking off and fragmenting.
4. Basilar skull fractures: These are fractures that occur at the base of the skull and can involve the brainstem or cranial nerves.
5. Cerebral edema: This is a condition in which fluid accumulates in the brain as a result of a head injury or other traumatic event.
6. Epidural hematoma: This is a collection of blood between the skull and the dura mater, which is the membrane that covers the brain.
7. Subdural hematoma: This is a collection of blood between the dura mater and the brain.
8. Intracerebral hematoma: This is a collection of blood within the brain tissue.

Skull fractures can be diagnosed using a variety of imaging tests, such as X-rays, CT scans, or MRI scans. Treatment for skull fractures may involve observation, medication, or surgery, depending on the severity of the injury and any other complications that may have arisen.

Complications of skull fractures can include:

1. Cerebral edema
2. Epidural hematoma
3. Subdural hematoma
4. Intracerebral hematoma
5. Brain contusion
6. Skull base fractures
7. Facial trauma
8. Sinus fractures
9. Orbital blowout fractures
10. Meningitis or sepsis.

It is important to seek medical attention immediately if any of the following symptoms are present:

1. Severe headache
2. Confusion or disorientation
3. Slurred speech or difficulty speaking
4. Weakness or numbness in the arms or legs
5. Vision changes, such as double vision or blurred vision
6. Difficulty with balance or coordination
7. Seizures or convulsions
8. Fever
9. Stiff neck
10. Loss of consciousness or coma.

1. Infection: Bacterial or viral infections can develop after surgery, potentially leading to sepsis or organ failure.
2. Adhesions: Scar tissue can form during the healing process, which can cause bowel obstruction, chronic pain, or other complications.
3. Wound complications: Incisional hernias, wound dehiscence (separation of the wound edges), and wound infections can occur.
4. Respiratory problems: Pneumonia, respiratory failure, and atelectasis (collapsed lung) can develop after surgery, particularly in older adults or those with pre-existing respiratory conditions.
5. Cardiovascular complications: Myocardial infarction (heart attack), cardiac arrhythmias, and cardiac failure can occur after surgery, especially in high-risk patients.
6. Renal (kidney) problems: Acute kidney injury or chronic kidney disease can develop postoperatively, particularly in patients with pre-existing renal impairment.
7. Neurological complications: Stroke, seizures, and neuropraxia (nerve damage) can occur after surgery, especially in patients with pre-existing neurological conditions.
8. Pulmonary embolism: Blood clots can form in the legs or lungs after surgery, potentially causing pulmonary embolism.
9. Anesthesia-related complications: Respiratory and cardiac complications can occur during anesthesia, including respiratory and cardiac arrest.
10. delayed healing: Wound healing may be delayed or impaired after surgery, particularly in patients with pre-existing medical conditions.

It is important for patients to be aware of these potential complications and to discuss any concerns with their surgeon and healthcare team before undergoing surgery.

Most nasopharyngeal neoplasms are rare and tend to affect children and young adults more frequently than older adults. The most common types of nasopharyngeal neoplasms include:

1. Nasopharyngeal carcinoma (NPC): This is the most common type of malignant nasopharyngeal neoplasm and tends to affect young adults in Southeast Asia more frequently than other populations.
2. Adenoid cystic carcinoma: This is a rare, slow-growing tumor that usually affects the nasopharynx and salivary glands.
3. Metastatic squamous cell carcinoma: This is a type of cancer that originates in another part of the body (usually the head and neck) and spreads to the nasopharynx.
4. Lymphoma: This is a type of cancer that affects the immune system and can occur in the nasopharynx.
5. Benign tumors: These include benign growths such as papillomas, fibromas, and meningiomas.

Symptoms of nasopharyngeal neoplasms can vary depending on the size and location of the tumor but may include:

* Difficulty swallowing
* Nosebleeds
* Headaches
* Facial pain or numbness
* Trouble breathing through the nose
* Hoarseness or voice changes
* Enlarged lymph nodes in the neck

Diagnosis of nasopharyngeal neoplasms usually involves a combination of imaging tests such as CT or MRI scans, endoscopy (insertion of a flexible tube with a camera into the nose and throat), and biopsy (removal of a small sample of tissue for examination under a microscope).

Treatment of nasopharyngeal neoplasms depends on the type, size, location, and stage of the tumor but may include:

* Surgery to remove the tumor
* Radiation therapy to kill cancer cells
* Chemotherapy to kill cancer cells
* Targeted therapy to attack specific molecules on cancer cells

Prognosis for nasopharyngeal neoplasms varies depending on the type and stage of the tumor but in general, early detection and treatment improve the chances of a successful outcome.

There are two main types of carotid stenosis:

1. Internal carotid artery stenosis: This type of stenosis occurs when the internal carotid artery, which supplies blood to the brain, becomes narrowed or blocked.
2. Common carotid artery stenosis: This type of stenosis occurs when the common carotid artery, which supplies blood to the head and neck, becomes narrowed or blocked.

The symptoms of carotid stenosis can vary depending on the severity of the blockage and the extent of the affected area. Some common symptoms include:

* Dizziness or lightheadedness
* Vertigo (a feeling of spinning)
* Blurred vision or double vision
* Memory loss or confusion
* Slurred speech
* Weakness or numbness in the face, arm, or leg on one side of the body

If left untreated, carotid stenosis can lead to a stroke or other serious complications. Treatment options for carotid stenosis include medications to lower cholesterol and blood pressure, as well as surgical procedures such as endarterectomy (removing plaque from the artery) or stenting (placing a small mesh tube in the artery to keep it open).

In conclusion, carotid stenosis is a serious medical condition that can lead to stroke and other complications if left untreated. It is important to seek medical attention if symptoms persist or worsen over time.

This type of hearing loss cannot be treated with medication or surgery, and it is usually permanent. However, there are various assistive devices and technology available to help individuals with sensorineural hearing loss communicate more effectively, such as hearing aids, cochlear implants, and FM systems.

There are several causes of sensorineural hearing loss, including:

1. Exposure to loud noises: Prolonged exposure to loud noises can damage the hair cells in the inner ear and cause permanent hearing loss.
2. Age: Sensorineural hearing loss is a common condition that affects many people as they age. It is estimated that one-third of people between the ages of 65 and 74 have some degree of hearing loss, and nearly half of those over the age of 75 have significant hearing loss.
3. Genetics: Some cases of sensorineural hearing loss are inherited and run in families.
4. Viral infections: Certain viral infections, such as meningitis or encephalitis, can damage the inner ear and cause permanent hearing loss.
5. Trauma to the head or ear: A head injury or a traumatic injury to the ear can cause sensorineural hearing loss.
6. Tumors: Certain types of tumors, such as acoustic neuroma, can cause sensorineural hearing loss by affecting the auditory nerve.
7. Ototoxicity: Certain medications, such as certain antibiotics, chemotherapy drugs, and aspirin at high doses, can be harmful to the inner ear and cause permanent hearing loss.

It is important to note that sensorineural hearing loss cannot be cured, but there are many resources available to help individuals with this condition communicate more effectively and improve their quality of life.

1. Strabismus (crossed eyes): A condition in which the eyes do not align properly and point in different directions.
2. Esotropia (crossed eyes): A condition in which one or both eyes turn inward.
3. Exotropia (wide-eyed): A condition in which one or both eyes turn outward.
4. Hypertropia (upward-pointing eyes): A condition in which one or both eyes elevate excessively.
5. Hypotropia (downward-pointing eyes): A condition in which one or both eyes lower excessively.
6. Diplopia (double vision): A condition in which two images of the same object are seen due to improper alignment of the eyes.
7. Nystagmus (involuntary eye movements): A condition characterized by rapid, involuntary movements of the eyes.
8. Ocular flutter: A condition characterized by small, rapid movements of the eyes.
9. Progressive supranuclear palsy (PSP): A rare degenerative disorder that affects movement and causes difficulty with eye movements.
10. Parkinson's disease: A neurodegenerative disorder that can cause eye movements to be slow, stiff, or irregular.

These disorders can have a significant impact on an individual's quality of life, affecting their ability to perform daily tasks, read, drive, and participate in social activities. Treatment options vary depending on the specific condition and may include glasses or contact lenses, prism lenses, eye exercises, and surgery. In some cases, medications such as anticholinergic drugs or botulinum toxin injections may be used to help improve eye movements.

Cranial nerve mnemonics Standring, Susan; Borley, Neil R. (2008). "Overview of cranial nerves and cranial nerve nuclei". Gray's ... Some considered there to be thirteen pairs of cranial nerves, including cranial nerve zero. The numbering of the cranial nerves ... The nerves are: the olfactory nerve (I), the optic nerve (II), oculomotor nerve (III), trochlear nerve (IV), trigeminal nerve ( ... glossopharyngeal nerve (IX), vagus nerve (X), accessory nerve (XI), and the hypoglossal nerve (XII). Cranial nerves are ...
"Cranial Nerves - Function, Table, Anatomy and FAQs". Butler, Ann B.; Hodos, William (2005). Comparative Vertebrate Neuroanatomy ... ISBN 1-58663-097-0. (Articles with short description, Short description matches Wikidata, Cranial nerves). ...
Is the cranial accessory nerve really a portion of the accessory nerve? Anatomy of the cranial nerves in the jugular foramen. ... The cranial root of accessory nerve (or part) is the smaller of the two portions of the accessory nerve. It is generally ... part of the vagus nerve and not part of the accessory nerve proper because the cranial component rapidly joins the vagus nerve ... it appears more useful to consider them in general to be part of the cranial roots of the vagal nerve. The accessory nerve ...
... in the brain stem that is associated with one or more of the cranial nerves. Axons carrying information to and from the cranial ... A cranial nerve nucleus is a collection of neurons (gray matter) ... ISBN 0-387-94227-0 Nuclei of origin of cranial motor nerves ... Primary terminal nuclei of the afferent (sensory) cranial nerves schematically represented; lateral view. Brain stem sagittal ... All the nuclei except that of the trochlear nerve (CN IV) supply nerves of the same side of the body. In general, motor nuclei ...
... the cranial nerve ganglia are ganglia of certain cranial nerves. They can be parasympathetic or sensory. All cranial nerve ... Cranial Nerve 8 (Vestibulocochlear)". Neuroanatomy, Cranial Nerve 8 (Vestibulocochlear) on U.S. National Library of Medicine. ... nerve inferior ganglion of glossopharyngeal nerve superior ganglion of vagus nerve inferior ganglion of vagus nerve Purves, ... The four cranial parasympathetic ganglia are: ciliary ganglion pterygopalatine ganglion otic ganglion submandibular ganglion ...
The facial nerve is the seventh of 12 cranial nerves. This cranial nerve controls the muscles in the face. Facial nerve palsy ... Eyes Oculomotor nerve palsy - Oculomotor nerve (III) Fourth nerve palsy - Trochlear nerve (IV) Sixth nerve palsy - Abducens ... Cranial nerve disease is an impaired functioning of one of the twelve cranial nerves. Although it could theoretically be ... It is possible for a disorder of more than one cranial nerve to occur at the same time, if a trauma occurs at a location where ...
The cranial nerve exam is a type of neurological examination. It is used to identify problems with the cranial nerves by ... "Examination of the Cranial Nerves". 22 April 2016. "Examination of the Cranial Nerves". NeurologyExam.com Free neurology exam ... Cranial nerves Cranial nerve nucleus Jon Brillman; Scott Kahan (1 March 2005). In A Page Neurology. Lippincott Williams & ... Each test is designed to assess the status of one or more of the twelve cranial nerves (I-XII). These components correspond to ...
The cranial nerve nuclei schematically represented; dorsal view. Motor nuclei in red; sensory in blue. Nuclei of origin of ... cranial motor nerves schematically represented; lateral view. Primary terminal nuclei of the afferent (sensory) cranial nerves ... Joyce, Christopher; Le, Patrick H.; Peterson, Diana C. (2022), "Neuroanatomy, Cranial Nerve 3 (Oculomotor)", StatPearls, ... Cranial Nerves. Yale School of Medicine. Archived from the original on 2016-03-03. (CS1 maint: multiple names: authors list, ...
Cranial Nerves. Yale School of Medicine. Archived from the original on 2016-03-03. Table at doctor_uae Portal: Anatomy v t e ( ... and joins with the greater petrosal nerve to form the nerve of the pterygoid canal, also known as the Vidian nerve. The deep ... The deep petrosal nerve is a branch of the internal carotid plexus which runs through the carotid canal lateral to the internal ... The neuron cell bodies of the axons which form the deep petrosal nerve are found in the superior cervical ganglion. Sympathetic ...
Cranial Nerves. Yale School of Medicine. Archived from the original on 2016-03-03. cranialnerves at The Anatomy Lesson by ... The pharyngeal branch of the vagus nerve, the principal motor nerve of the pharynx, arises from the upper part of the ganglion ... and consists principally of filaments from the cranial portion of the accessory nerve. It passes across the internal carotid ... which is supplied by the nerve to tensor veli palatini, a branch of the nerve to medial pterygoid (which itself is a branch of ...
... diagram cranialnerves at The Anatomy Lesson by Wesley Norman (Georgetown University) (IX) lesson3 at The ... The nucleus of the lesser petrosal nerve is the inferior salivatory nucleus. Tympanic nerve Glossopharyngeal Nerve, Overview of ... Sep 2007). "The course of the lesser petrosal nerve on the middle cranial fossa". Neurosurgery. 61 (3 Suppl): 15-23. doi: ... The lesser petrosal nerve (also known as the small superficial petrosal nerve) is the general visceral efferent (GVE) component ...
The information is sent via the olfactory nerve (Cranial Nerve I) to the olfactory bulb. After the processing in the bulb the ... "1-4". Cranial Nerves. Yale School of Medicine. Archived from the original on 2016-03-03. NIF Search - Anterior Olfactory ...
Cranial Nerves. Yale School of Medicine. Archived from the original on 2016-03-03. (Articles with short description, Short ... The olfactory tract is a bilateral bundle of afferent nerve fibers from the mitral and tufted cells of the olfactory bulb that ...
... cranialnerves at The Anatomy Lesson by Wesley Norman (Georgetown University) (IX) "9-13". Cranial Nerves. Yale School of ... The carotid branch of the glossopharyngeal nerve (carotid sinus nerve or Hering's nerve) is a small branch of the ... It communicates with the vagus nerve and sympathetic trunk and then divides in the angle of the bifurcation of the common ... It is a branch of the glossopharyngeal nerve that runs downwards, anterior to the internal carotid artery. ...
The vagus nerve, also known as the tenth cranial nerve, cranial nerve X, or simply CN X, is a cranial nerve that interfaces ... Pharyngeal nerve Superior laryngeal nerve Superior cervical cardiac branches of vagus nerve Inferior cervical cardiac branch ... Cranial Nerves. Yale School of Medicine. Archived from the original on 3 March 2016. cranialnerves at The Anatomy Lesson by ... Cranial Nerve XI: The Spinal Accessory Nerve. NCBI Bookshelf. Butterworths. ISBN 9780409900774. PMID 21250228. Retrieved 30 May ...
The olfactory nerve, also known as the first cranial nerve, cranial nerve I, or simply CN I, is a cranial nerve that contains ... The olfactory nerve is the shortest of the twelve cranial nerves and, similar to the optic nerve, does not emanate from the ... the olfactory nerve is somewhat unusual among cranial nerves because it is capable of some regeneration if damaged. The ... Cranial Nerves. Yale School of Medicine. Archived from the original on 2016-03-03. cranialnerves at The Anatomy Lesson by ...
This syndrome occurs when the sixth cranial nerve which controls the lateral rectus muscle does not develop properly. It is ... Anatomy figure: 29:01-05 at Human Anatomy Online, SUNY Downstate Medical Center "6-1". Cranial Nerves. Yale School of Medicine ... A sixth nerve palsy, also known as abducens nerve palsy, is a neurological defect that results from a damaged or impaired ... Nguyen, Van; Varacallo, Matthew (2019), "Neuroanatomy, Cranial Nerve 6 (Abducens)", StatPearls, StatPearls Publishing, PMID ...
ISBN 978-961-90305-5-4. "1-5". Cranial Nerves. Yale School of Medicine. Archived from the original on 2016-03-03. v t e (All ...
It joins the facial nerve (cranial nerve VII) inside the facial canal, at the level where the facial nerve exits the skull via ... The chorda tympani carries two types of nerve fibers from their origin with the facial nerve to the lingual nerve that carries ... with each nerve acting to inhibit the signals of other nerves. The chorda tympani exits the cranial cavity through the internal ... The chorda tympani is part of one of three cranial nerves that are involved in taste. The taste system involves a complicated ...
doi:10.1016/B978-0-7216-9491-7.50012-0. ISBN 978-0-7216-9491-7. "3-11". Cranial Nerves. Yale School of Medicine. Archived from ... The short ciliary nerves are nerves of the orbit around the eye. They are branches of the ciliary ganglion. They supply ... Damage to the short ciliary nerve may result in loss of the pupillary light reflex, or mydriasis. The short ciliary nerves are ... The short ciliary nerves contain both parasympathetic and sympathetic nerve fibers. The parasympathetic fibers arise from the ...
The accessory nerve, also known as the eleventh cranial nerve, cranial nerve XI, or simply CN XI, is a cranial nerve that ... "Is the cranial accessory nerve really a portion of the accessory nerve? Anatomy of the cranial nerves in the jugular foramen". ... The spinal accessory nerve is notable for being the only cranial nerve to both enter and exit the skull. This is due to it ... The cranial component rapidly joins the vagus nerve, and there is ongoing debate about whether the cranial part should be ...
... on Cranial Nerves; Selected Letters trans. John Reddick (Penguin Classics, 1993) ISBN 0-14-044586-2. Georg Büchner, Danton's ...
Love, Russell J.; Webb, Wanda G. (1992). "7 - The Cranial Nerves". Neurology for the Speech-Language Pathologist (2nd ed.). ... The vertical of the tongue is supplied by the hypoglossal nerve (CN XII). The vertical muscle of the tongue moves the tongue. ...
The hypoglossal nerve, also known as the twelfth cranial nerve, cranial nerve XII, or simply CN XII, is a cranial nerve that ... The hypoglossal nerve is one of twelve cranial nerves found in amniotes including reptiles, mammals and birds. As with humans, ... The hypoglossal nerve may be connected (anastomosed) to the facial nerve to attempt to restore function when the facial nerve ... Because of the close proximity of the nerve to other structures including nerves, arteries, and veins, it is rare for the nerve ...
Love, Russell J.; Webb, Wanda G. (1992). "7 - The Cranial Nerves". Neurology for the Speech-Language Pathologist (2nd ed.). ... The inferior longitudinal muscle of the tongue is supplied by the hypoglossal nerve (CN XII). The inferior longitudinal muscle ...
... Trigeminal nerve Cranial nerves This article incorporates text in the public domain from page 889 of the 20th ... alveolar nerve Infraorbital nerve Inferior palpebral nerve Superior labial nerve Lateral nasal nerve The Maxillary nerve gives ... Middle meningeal nerve in the meninges Zygomatic nerve (zygomaticotemporal nerve, zygomaticofacial nerve), through the Inferior ... is one of the three branches or divisions of the trigeminal nerve, the fifth (CN V) cranial nerve. It comprises the principal ...
... also known as the second cranial nerve, cranial nerve II, or simply CN II, is a paired cranial nerve that transmits visual ... Optic nerve Optic nerve Human brain dura mater (reflections) Optic nerve Optic nerve Optic nerve Cerebrum.Inferior view.Deep ... Cranial nerve Vilensky, Joel; Robertson, Wendy; Suarez-Quian, Carlos (2015). The Clinical Anatomy of the Cranial Nerves: The ... The optic nerve has been classified as the second of twelve paired cranial nerves, but it is technically part of the central ...
Love, Russell J.; Webb, Wanda G. (1992). "7 - The Cranial Nerves". Neurology for the Speech-Language Pathologist (2nd ed.). ... It is supplied by the hypoglossal nerve (CN XII). It moves the tongue. The transverse muscle of the tongue is an intrinsic ... The transverse lingual muscle is supplied by the hypoglossal nerve (CN XII). The transverse muscle of the tongue muscle moves ...
Spinal nerves. Spinal cord and vertebral canal. Deep dissection. Cranial nerves "Spinal Nerves". National Library of Medicine. ... five pairs of lumbar nerves, five pairs of sacral nerves, and one pair of coccygeal nerves. The spinal nerves are part of the ... The fourth nerve is named the furcal nerve, from the fact that it is subdivided between the two plexuses. The sacral nerves are ... Each spinal nerve is a mixed nerve, formed from the combination of nerve fibers from its dorsal and ventral roots. The dorsal ...
Love, Russell J.; Webb, Wanda G. (1992). "7 - The Cranial Nerves". Neurology for the Speech-Language Pathologist (2nd ed.). ... The superior longitudinal muscle of the tongue is supplied by the hypoglossal nerve (CN XII). The superior longitudinal muscle ...
There is nearly complete fusion of several cranial bones, namely the nasal and frontal bones; they are poorly differentiated ... Eumegamyines additionally typically have a well developed stylomastoid foramen, which funnels the facial nerve, and a short ear ... Cox, Philip G.; Rinderknecht, Andrés; Blanco, R. Ernesto (2015). "Predicting bite force and cranial biomechanics in the largest ... which contains the optic nerve and ophthalmic artery, corresponding to vision. Eumegamyines typically feature an unusual large ...
Abdel-Kader, T.G.; Ali, R.S.; Ibrahim, N.M. (2011). "The Cranial Nerves of Mabuya quinquetaeniata III: Nervus Trigeminus" (PDF ... All of these canals would have brought nerves and nutrient-rich tissue to the root of the canines and the rest of the upper jaw ... The forward-directed canal also splits into the three main branches of the infraorbital nerve, all of which connect to the ... CT scanning shows that these openings lead to canals that connect to the trigeminal nerve, which controls facial sensitivity. ...
Thus, tumor growth can impinge nerve function and result in vision loss and diplopia. As the tumor metastasizes to the oral ... Due to the location of the tumor and its proximity to the cranial cavity, esthesioneuroblastoma can be highly invasive and ... Most studies reported cranial surgical resection with radiotherapy or chemotherapy to target the tumor. Esthesioneuroblastoma ... Esthesioneuroblastoma occurs in the upper nasal cavity, near the optic nerves and optic chiasm. ...
It transmits the facial (VII) and vestibulocochlear (VIII) cranial nerves into a canal in the petrous temporal bone. Lies ... Animation Posterior cranial fossa at human fetus Base of skull Posterior cranial fossa Posterior cranial fossa A tumor of the ... Anterior cranial fossa Middle cranial fossa Wikimedia Commons has media related to Posterior cranial fossa. Anatomy photo:22:os ... The posterior cranial fossa is part of the cranial cavity, located between the foramen magnum and tentorium cerebelli. It ...
... silvery-blonde scalp hair Cranial nerve paralysis, moyamoya disease, stroke, intellectual disability Treatment of Roberts ...
97%: Facial paralysis, as Roosevelt had, in the absence of other cranial nerve abnormalities, is not consistent with a polio ... since polioviruses do not attack autonomic nerves, but are common in GBS. 93%: Meningismus (neck stiffness), a characteristic ...
The scalp is innervated by motor nerves and sensory nerves. The trigeminal nerve (CNV) is one of the important cranial sensory ... nerve and Supraorbital nerve Zygomaticotemporal nerve Auriculotemporal nerve Lesser occipital nerve Greater occipital nerve ... All large blood vessels and nerves of the scalp are located in this layer. The next layer is the galea Aponeurotica, which ... The large blood vessels and nerves of the scalp don't pierce this layer.Loose connective tissue between the periosteum and the ...
The incisura has also a close relationship with the first 6 cranial nerves. If intracranial pressure superior to the cerebellar ...
Cranial neuritis is an inflammation of cranial nerves. When due to Lyme it most typically causes facial palsy, impairing ... The spirochetes may also induce host cells to secrete quinolinic acid, which stimulates the NMDA receptor on nerve cells, which ... Lyme radiculopathy is an inflammation of spinal nerve roots that often causes pain and less often weakness, numbness, or ... In infection involving the PNS, electromyography and nerve conduction studies can be used to monitor objectively the response ...
... and possibly lower cranial nerve (IX, X, XI, XII) deficits, tetraparesis or abnormal breathing. Among these, cranial nerve ... Surgery may become necessary if there is significant compression of the brainstem, spinal cord, the lower cranial nerves or ... Orbay T, Aykol S, Seçkin Z, Ergün R (May 1989). "Late hypoglossal nerve palsy following fracture of the occipital condyle". ...
... cranial or spinal nerves) or lower motor neuron system. Depending on which nerves are damaged, flaccid dysarthria affects ... If the muscles of the face are affected (i.e. if there is damage to cranial nerve VII; V for the jaw in mastication), there may ... Flaccid dysarthria is caused when damage occurs to the motor unit (one or more cranial or spinal nerves). Processes that can ... Some common signs include the following Phonation and prosody: Damage to cranial nerve X can present as changes in voice ...
... as studies have shown that bacteria in the gut can activate stress response through the vagus nerve, a cranial nerve ...
Cranial nerve nuclei, Medulla oblongata, Vagus nerve, Glossopharyngeal nerve, Facial nerve). ... Primary terminal nuclei of the afferent (sensory) cranial nerves schematically represented; lateral view. Solitary tract Duane ... doi:10.1038/s41586-022-05139-5. Dulak, Dominika; Naqvi, Imama A (2020). Neuroanatomy, Cranial Nerve 7 (Facial). StatPearls. ... Taste information from the facial nerve via the chorda tympani (anterior 2/3 of the tongue), glossopharyngeal nerve (posterior ...
Cranial and caudal, which describe structures close to the top of the skull (cranial), and towards the bottom of the body ( ... A plexus refers to a net-like arrangement of a nerve. The term anatomical variation is used to refer to a difference in ...
... "inferior maxillary nerve", enters infratemporal fossa from the middle cranial fossa through the foramen ovale of the sphenoid ... meningeal nerve buccal nerve auriculotemporal nerve lingual nerve inferior alveolar nerve auricle external acoustic meatus ... The infratemporal fossa contains the mandibular nerve, the inferior alveolar nerve, the lingual nerve, the buccal nerve, the ... These are the masseteric nerve to masseter muscle, the deep temporal nerve to temporalis muscle, the lateral pterygoid nerve to ...
The cranial nerves containing SVA fibers are the olfactory nerve (I), the facial nerve (VII), the glossopharyngeal nerve (IX), ... and the vagus nerve (X). The facial nerve receives taste from the anterior 2/3 of the tongue; the glossopharyngeal from the ... posterior 1/3, and the vagus nerve from the epiglottis. The sensory processes, using their primary cell bodies from the ...
... thrombosis and nerve compression syndrome of cranial nerves XI and XII. One death case appeared in the scientific literature, ... which in turn triggers autoimmunity and degeneration of the nerve's myelin sheath. While the initial article on CCSVI claimed ...
The vagus nerve provides efferent nervous signals out from the hunger and satiety centers of the hypothalamus, a region of the ... is sometimes injured by ongoing treatment for acute lymphoblastic leukemia or surgery or radiation to treat posterior cranial ... "Could nerve-snipping spur weight loss? - USATODAY.com". USA Today. 2007-07-02. Retrieved 2010-05-27. Lustig, Robert H.; Pamela ... A vagotomy is a surgical procedure that involves removing part of the vagus nerve. A plain vagotomy eliminates the ...
The hypoglossal nerve plays an important role in controlling movements of the tongue. In 1998, a research team used the size of ... It is necessary to take into consideration the skull base, the mandible, the cervical vertebrae and a cranial reference plane. ... Subsequently, however, it was demonstrated that hypoglossal canal size and nerve sizes are not correlated, and it is now ... the hypoglossal canal in the base of fossil skulls in an attempt to estimate the relative number of nerve fibres, claiming on ...
Damage to peripheral nerves may impair sensation, movement, gland, or organ function depending on which nerves are affected; in ... Brachial neuritis Cranial neuritis such as Bell's palsy Optic neuritis Vestibular neuritis Wartenberg's migratory sensory ... These nerves are not under a person's conscious control and function automatically. Autonomic nerve fibers form large ... In cases of polyneuropathy, many nerve cells in various parts of the body are affected, without regard to the nerve through ...
... damage to the inferior alveolar nerve occurs in 3.5% of mandibular distraction, tooth bud injury in 2%, and facial nerve injury ... Introduction of Multidirectional Cranial Distraction Osteogenesis". Journal of Korean Neurosurgical Society. 59 (3): 233-41. ...
... cranial nerve assessment (CNI-CNXII), MMSE) Ordering X-rays and requesting further investigations Interpreting X-ray and blood ...
"The foramen is probed with a nerve hook to ensure that the nerve is free". To decompress a longer part of the cervical canal a ... The extent of the slot should not exceed half of the vertebral body - cranial or caudal, but at the same time is providing more ... Attention is paid on any deep nerve structures as the recurrent laryngeal nerve. The goal is to expose the affected disc and ... Alternatively, if only a single nerve root is affected it is also possible to release the compressed nerve root via a ...
Dizziness Loss of sensation Cranial nerve disturbance Loss of the ability to know how joints are positioned Lhermitte's sign (' ... Symptoms vary depending on whether the spinal cord, brain stem, nerves, or blood supply is affected by the pressure.[citation ...
He was a student of François Magendie in Paris from 1824-1825, composing a thesis on cranial nerves, after which he studied ...
However, the way in which the bone was laid down makes it possible to examine the imprints of nerves and other soft tissues. ... This is due to the development of paired fins, and their complicated cranial anatomy. The osteostracans were more similar to ...
Cranial nerve nuclei, All stub articles, Neuroanatomy stubs). ... which joins with the cochlear nerve. It receives its blood ... Vestibular nerve Vestibular system This article incorporates text in the public domain from the 20th edition of Gray's Anatomy ...
All of which are present in the cortex, medulla, hippocampus, and motor cells of the spinal cord and XIIth cranial nerve. In ...
Cranial nerve palsies occur in some unusual cases. In the bestselling 1996 non-fiction book Into Thin Air: A Personal Account ... but the increase in brain volume from edema does not likely cause cranial vault impingement. The presence of large sulci ...
Cranial Nerve I  Component: Sensory  Function: Smell  Origin: Olfactory receptor nerve cells  Opening to the Skul... ... Cranial Nerve I * Component: Sensory  Function: Smell  Origin: Olfactory receptor nerve cells  Opening to the Skull: ... Cranial Nerve VI * Component: Motor  Function: Lateral rectus muscle turns eyeball laterally  Origin: Anterior Surface of ... Cranial Nerve III * Component: Motor  Function:  Raises upper eyelid  Turns eyeball upward, downward and medially  ...
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... cranial nerve [CN] X) is the longest cranial nerve in the body, containing both motor and sensory functions in both the ... Because of the wide distribution of the nerve throughout the body, there are several clinical correlations of the vagus nerve. ... The nerve travels widely throughout the body affecting several organ systems and regions of the body, such as the tongue, ... Neuroanatomy, Cranial Nerve 10 (Vagus Nerve) - StatPearls. Neuroanatomy, Cranial Nerve 10 (Vagus Nerve) - StatPearls. ...
... imaging of the cranial nerves (CNs) was difficult, and mass lesions arising from these nerves was often indirectly detected ... encoded search term (Cranial Nerve Schwannoma Imaging) and Cranial Nerve Schwannoma Imaging What to Read Next on Medscape ... Rare Cranial Nerve Schwannomas: A Retrospective Review of Nontrigeminal, Nonvestibular Cranial Nerve Schwannomas. J Neurosci ... Transient ocular motor nerve palsies associated with presumed cranial nerve schwannomas. J Neuroophthalmol. 2015 Jun. 35 (2): ...
The cranial nerve experts at UC San Diego Health diagnose and treat conditions involving the 12 pairs of cranial nerves that ... Treatment for Cranial Nerve Disorders. Treatment varies depending on the condition. Our cranial nerve specialists provide ... Cranial nerve disorders can cause symptoms that are challenging to live with on a day-to-day basis. ... The experts at UC San Diego Health diagnose and treat conditions involving the 12 pairs of cranial nerves that originate from ...
This lesson provides helpful information on Cranial Nerves in the context of Brain to help students study for a college level ... Cranial Nerves. Cranial nerves exit the inferior region of the cranium through foramina in the cranial bones and are numbered I ... Name and Function of Cranial Nerves. Number. Name. Function. I. Olfactory nerves. These nerves help produce the sense of smell. ... Cranial nerve I is located closest to the frontal lobe, while cranial nerve XII is found closest to the brain stem.. Locations ...
1 Abstracts with Cranial Nerve Diseases Research. Filter by Study Type. Human Study. ... Diseases : Arnold-Chiari Malformation, Chiaris Syndrome, Cranial Nerve Diseases. Therapeutic Actions : Applied Kinesiology, ... 2 Therapeutic Actions Researched for Cranial Nerve Diseases Name. AC. CK. Focus. ... kinesiology chiropractic treatment may have therapeutic value in symptomatic Arnold-Chiari malformation and cranial nerve ...
Because cranial nerves determine and modulate brain function, invasive and non-invasive cranial nerve electrical … ... The cranial nerves are the pathways through which environmental information (sensation) is directly communicated to the brain, ... Bottom row: Cranial nerves overlaid with cranial nerve stimulation montages (trigeminal: orange, vagus: green, vestibular: ... Because cranial nerves determine and modulate brain function, invasive and non-invasive cranial nerve electrical stimulation ...
Dissection of the internal carotid artery causing Horner syndrome and palsy of cranial nerve XII. Neema Kasravi, Andrew Leung, ... Dissection of the internal carotid artery causing Horner syndrome and palsy of cranial nerve XII. CMAJ 2010;182:E373-E377. ... Dissection of the internal carotid artery causing Horner syndrome and palsy of cranial nerve XII ... Dissection of the internal carotid artery causing Horner syndrome and palsy of cranial nerve XII ...
A 25-month-old female child presented with a cranial neuropathy, a spastic … ... Isolated cranial nerve enhancement in metachromatic leukodystrophy Rani K Singh 1 , Robert T Leshner, Nadja Kadom, Adeline L ... Isolated cranial nerve enhancement in metachromatic leukodystrophy Rani K Singh et al. Pediatr Neurol. 2009 May. ... Multiple cranial nerve enhancement: a new MR imaging finding in metachromatic leukodystrophy. Maia AC Jr, da Rocha AJ, da Silva ...
Cranial nerve demyelination was found during autopsy. Bilateral, asymmetric clinical signs, although rare, do not rule out ... Postmortem cranial nerve tissue from a patient with botulism. A) Fragmentation of myelin sheaths and inflammatory infiltration ... Postmortem cranial nerve tissue from a patient with botulism. A) Fragmentation of myelin sheaths and inflammatory infiltration ... Asymmetric Type F Botulism with Cranial Nerve Demyelination. Emerging Infectious Diseases. 2012;18(1):102-104. doi:10.3201/ ...
Overview of Neuro-ophthalmologic and Cranial Nerve Disorders - Etiology, pathophysiology, symptoms, signs, diagnosis & ... Examination of the cranial nerves How to Assess the Cranial Nerves (See also Neuro-ophthalmologic and Cranial Nerve Disorders ... See also How to Assess the Cranial Nerves How to Assess the Cranial Nerves (See also Neuro-ophthalmologic and Cranial Nerve ... see table Cranial Nerves Cranial Nerves ). One or more cranial nerves may be affected. ...
Cavernous sinus and jugular thromboses, base of skull osteomyelitis and cranial nerve palsies: catastrophic complications of ... Cavernous sinus and jugular thromboses, base of skull osteomyelitis and cranial nerve palsies: catastrophic complications of ...
Dysphagia and disrupted cranial nerve development in a mouse model of DiGeorge (22q11) deletion syndrome Beverly A. Karpinski, ... There is currently no evaluation of disrupted oro-facial development, hindbrain gene expression or cranial nerve (CN) ... Tbx1 mutation causes multiple cardiovascular defects and disrupts neural crest and cranial nerve migratory pathways ... Dysphagia and disrupted cranial nerve development in a mouse model of DiGeorge (22q11) deletion syndrome. Dis Model Mech 1 ...
... with the facial nerve being the most susceptible to injury of all the cranial nerves. Damage to the facial nerve can result in ... A cranial nerve is any of the 12 paired nerves that arise from the lower surface of the brain, with one of each pair on each ... All other nerves in the body run from the spinal cord. There are many causes for cranial nerve injury, including head trauma or ... The cranial nerves are the only nerves that run directly from the brain, and they control all facial and throat movements, ...
The glossopharyngeal nerve is the 9th cranial nerve (CN IX). It is one of the four cranial nerves that has sensory, motor, and ... During a carotid endarterectomy, injury can occur to different cranial nerves, including the glossopharyngeal nerve (although ... Tympanic nerve (AKA nerve of Jacobson) - carries parasympathetic fibers and eventually becomes the lesser petrosal nerve, ... and a communicating branch to cranial nerve X (vagus nerve). Special visceral efferent fibers (branchial motor) are the main ...
Start Over You searched for: Subjects Cranial Nerves ✖Remove constraint Subjects: Cranial Nerves Publication Year 1890 to 1899 ... Cranial Nerves Archival Collection: The Florence R. Sabin Papers (Profiles in Science) 2. Some practical observations upon the ... Some practical observations upon the origin, communications, and distributions of the cranial nerves1 ... and distributions of the cranial nerves Author(s): Allison, Charles C., author Publication: [Omaha] : [publisher not identified ...
The 12 cranial nerves include the: olfactory nerve optic nerve oculomotor nerve trochlear nerve trigeminal nerve abducens nerve ... An Easy Way to Learn the Cranial Nerves. Cranial Nerves. Test one ear at a time. Students found cranial nerve very difficult to ... Cranial nerve tricks to remember , List of cranial nerves made easy - This lecture explains easy cranial nerve mnemonics. ... cranial nerves. Diagram of Cranial Nerves. to get us started, a quick survey on the Cranial Nerves. Total Points. Ooh, ooh, ooh ...
The Cranial Nerves that control the eye include the Optic Nerve, the Oculomotor Nerve, the Trochlear Nerve and the Abducens ... a left and a right nerve for each. Some control muscles and others control senses. ... There are 12 pairs of nerves in the Central Nerve System, ... Cranial Nerve 2 (CN II) - Optic Nerve: Vision. CNII (Cranial ... Cranial Nerve 4 (CN IV) - Trochlear Nerve: Muscles for Eye Movement CN IV (Cranial Nerve 4), controls eyeball movement, ...
Optic Nerve (Cranial Nerve II). The optic nerve (n. opticus), or second cranial nerve, is actually a tract of the brain and not ... Trigeminal Nerve (Cranial Nerve V). The trigeminal nerve (n. trigeminus), or fifth cranial nerve, has both motor and sensory ... Oculomotor Nerve (Cranial Nerve III). The oculomotor nerve (n. oculomotorius), or third cranial nerve, consists primarily of ... Trochlear Nerve (Cranial Nerve IV). The trochlear nerve (n. trochlearis), or fourth cranial nerve, provides general somatic ...
Lotery, A, Moore, J & Johnston, PB 1994, Review of 3rd, 4th and 6th Cranial Nerve Palsies, IRISH JOURNAL OF MEDICAL SCIENCE, ... Review of 3rd, 4th and 6th Cranial Nerve Palsies. In: IRISH JOURNAL OF MEDICAL SCIENCE. 1994 ; Vol. 163, No. 1. pp. 0. ... Review of 3rd, 4th and 6th Cranial Nerve Palsies. / Lotery, A; Moore, Johnny; Johnston, PB. In: IRISH JOURNAL OF MEDICAL ... 1994). Review of 3rd, 4th and 6th Cranial Nerve Palsies. IRISH JOURNAL OF MEDICAL SCIENCE, 163(1), 0. ...
Cranial Nerves This module is a portion of a course on exploration of the nervous system. The focus of this module is on the ...
... has 12 cranial nerves, 5 of which are responsible/necessary for social engagement (a feeling of safety, restoration, resiliency ... and a desire to connect with others and not isolate). These five cranial nerves #5,7,9,10, & 11, manifest in our facial ... Cranial Nerves & Empathy. August 27, 2019. DIRT GROUP Global Leave a comment ... The autonomic nervous system (most commonly understood as the fight/flight and freeze system) has 12 cranial nerves, 5 of which ...
Cranial nerve examination. All cranial nerves are examined to rule out associated nerve involvement to help determine the site ... Optical coherence tomography (OCT) measurements of retinal nerve fiber layer (RNFL) provide an objective measurement of nerve ... Correlation of retinal nerve fiber layer thickness and visual fields in glaucoma: a broken stick model. Am J Ophthalmol. 2014 ... In optic nerve damage, the transmission of impulses to the occipital cortex is delayed. In patients with unilateral or markedly ...
Clinical Examination of the Cranial Nerves. Singh, Rohin; Pollock, Jordan R; Moore, M Lane; Lee, Yeonsoo S; Hudson, Miles; ...
Study Brain and Cranial Nerves flashcards. Play games, take quizzes, print and more with Easy Notecards. ...
The olfactory nerve or cranial nerve I is the shortest cranial nerve and is… ... Cranial nerves arise from the brain as 12 pairs and function as relays of information between the brain and other parts of the ... What Are the 12 Cranial Nerves and Their Functions?. ByTarik Regad November 24, 2021. December 15, 2022. ... Read More What Are the 12 Cranial Nerves and Their Functions?. ... What Is the Function of the Olfactory Nerve? ...
Oculomotor nerve= Eyeball and eyelid movements. V1 . Trochlear. = Eye movement. V. Trigeminal = This is the largest cranial ... nerves and it is divided into three branches, which consist of the ophthalmic, maxillary and mandibular nerves. The functions ... Olfactory nerve = Sense of smell. 11. Optic nerve = vision. 111. ... Leave a comment on The 12 cranial nerves. 1. Olfactory nerve = ... Hypoglossal nerve: This is the movement of tongue, swallowing and speech. The cranial nerves consist of 12 nerves that arise ...
The facial nerve is kinda like the Golden Retriever (sorry for the mixed cat and dog metaphors) of the Cranial Nerves. Easy ... Facial Nerve (Cranial Nerve VII) -- General Information , Iowa Head and Neck Protocols (uiowa.edu) ... It is one of the longest cranial nerves, originating in the brainstem and branching out throughout the face. The facial nerve ... we know that the facial nerve is involved. ... Fact Friday: CATaloging the Cranial Nerves - The Hypoglossal 12 ...
  • The oculomotor nerve, a somatic and visceral motor, when it is completely injured, results in the drooping of the upper eyelid, the inability of the pupil to constrict in light, and the inability to look up. (legalinfo.com)
  • Cranial nerves: facial nerve (inferior view) Finally, the oculomotor nerve, the trochlear nerve, the mandibular branch of the trigeminal nerve (V3), the abducens nerve, the facial nerve, the glossopharyngeal nerve, the vagus nerve, the spinal accessory nerve, and the hypoglossal nerve are responsible for motor functions. (theneurolight.com)
  • the Optic Nerve, the Oculomotor Nerve, the Trochlear Nerve and the Abducens Nerve. (nf2is.org)
  • 111. Oculomotor nerve= Eyeball and eyelid movements. (nursesyoumoveme.com)
  • They originate from the mesencephalon and also oculomotor nerve. (allhealthpost.com)
  • The third cranial nerve is called the oculomotor nerve . (aapos.org)
  • In the case of a cranial nerve that has multiple functions (such as the oculomotor nerve), it is possible for a palsy to affect all the various functions or only some of the functions of that nerve. (aapos.org)
  • Damage to the trochlear nerve, a somatic motor, travels the longest course of any cranial nerve within the skull, will cause an eye to rotate outwards. (legalinfo.com)
  • To assess the trochlear nerve, instruct the patient to follow your finger while you move it down toward his nose. (theneurolight.com)
  • Trochlear nerve (CN IV) Cranial nerve IV acts as a pulley to move the eyes downtoward the tip of the nose. (theneurolight.com)
  • Cranial nerve 4, the trochlear nerve, innervates the superior oblique muscle. (seekhealthz.com)
  • The fourth cranial nerve is called the trochlear nerve and the sixth cranial nerve is called the abducens nerve . (aapos.org)
  • The optic nerve is considered a special sensory, and can damage the sensation of vision. (legalinfo.com)
  • Optic nerve [II] Use this application as a pocket reference and memorize Cranial Nerves in a simple way. (theneurolight.com)
  • Some nerves that we usually think of as purely afferent or sensory, such as the optic nerve for vision, and the vestibulocochlear nerve, for hearing and balance, also have a small component of efferent motor neurons that can modify the function of the end organ by altering transmission at the receptor site. (veteriankey.com)
  • Color vision is more decreased in patients with optic nerve disorders than in those with retinal disorders, especially among individuals with ischemic and compressive optic neuropathy. (medscape.com)
  • This test measures the ability to perceive slight changes in luminance between regions that are not separated by definite borders and is a sensitive test for optic nerve function. (medscape.com)
  • It is a sensitive optic nerve function test. (medscape.com)
  • 11. Optic nerve = vision. (nursesyoumoveme.com)
  • The olfactory and optic nerve arise from the anterior portion of the brain called cerebrum. (nursesyoumoveme.com)
  • The second cranial nerve is called the optic nerve . (aapos.org)
  • Other cranial nerves include the olfactory nerve (smell), the optic nerve (vision) and the vestibulocochlear nerve (hearing and balance). (nih.gov)
  • CNS manifestations include vasculitis of small to medium-sized vessels of the brain or spinal cord and granulomatous masses that involve the orbit, optic nerve, meninges, or brain. (medscape.com)
  • For example, the olfactory nerve (I) relays smell, while the facial nerve (VII) relays motor functions to the face. (coursehero.com)
  • The olfactory nerve, a special sensory, can be particularly susceptible to injury, which results in a change in the sense of smell. (legalinfo.com)
  • Long after the present numbering scheme was adopted, an additional, accessory olfactory nerve, the nervus terminalis, was recognized as a vomeronasal component of the olfactory system and was sometimes given the numeral designation of cranial nerve "0. (veteriankey.com)
  • It is considered here as a part of cranial nerve I, the olfactory nerve. (veteriankey.com)
  • It was fusiform in shape and located within the cranial cavity on the ventrolateral surface of the olfactory bulb. (veteriankey.com)
  • 1. Olfactory nerve = Sense of smell. (nursesyoumoveme.com)
  • These are sensory nerves responsible for collecting the olfactory stimuli form the nose and carrying it to the human brain. (allhealthpost.com)
  • They originate from olfactory bulb cells and are the shortest of all the cranial nerves. (allhealthpost.com)
  • The vagus nerve (cranial nerve [CN] X) is the longest cranial nerve in the body, containing both motor and sensory functions in both the afferent and efferent regards. (nih.gov)
  • The pharyngeal nerve branches arise from the inferior ganglion of CN X containing both sensory and motor fibers. (nih.gov)
  • Cranial nerves can be sensory (carry sensory information to the brain), motor (carry motor information away from the brain), or mixed (carry both types of information).Cranial nerves are numbered I-XII (using Roman numerals), and their names describe either what they do or the structures they serve. (coursehero.com)
  • The trigeminal nerve is a somatic sensory and somatic motor nerve, which affect the sensation of touch to the face as well as the movement of the muscles in chewing. (legalinfo.com)
  • The auditory/vestibular nerve is a special sensory nerve and affects the sensation of hearing and balance. (legalinfo.com)
  • Then there is the glossopharyngeal nerve, which is a somatic and visceral motor nerve and a special and visceral sensory nerve. (legalinfo.com)
  • It is one of the four cranial nerves that has sensory, motor, and parasympathetic functions. (statpearls.com)
  • The glossopharyngeal nerve carries sensory, efferent motor, and parasympathetic fibers. (statpearls.com)
  • After the jugular foramen are the superior and inferior ganglia, which house the cell bodies of the sensory fibers, and then the nerve descends the neck, where it provides innervation to the stylopharyngeus and sensation to the carotid sinus and body. (statpearls.com)
  • Either way, they can be helpful for remembering the names of the twelve cranial nerves , as well as remembering which nerves are sensory, motor, or both. (theneurolight.com)
  • Some of the primarily motor cranial nerves probably have general proprioceptive sensory neurons as well. (veteriankey.com)
  • X . Vagus nerve = Smooth muscle sensory and motor control in the throat, lung, heart and the digestive system. (nursesyoumoveme.com)
  • These nerves are responsible for connecting the brain to different body parts such as organs, muscles, motors and sensory organs. (allhealthpost.com)
  • They are a part of the peripheral nervous system and relate the cervical and cranial structures of the brain in an afferent direction and the vegetative, sensorial, motor and sensory structures in an efferent direction. (allhealthpost.com)
  • Trigeminal happens to be a mixed cranial nerve consisting of the motor, sensory and sensitive nerves. (allhealthpost.com)
  • The vestibule cochlear happens to be a sensory nerve. (allhealthpost.com)
  • This way, they will have a deep understanding of the types of cranial nerve injuries, and they will be well versed in your state's statutes and laws relative to personal injury, cranial nerve injury, and medical malpractice. (legalinfo.com)
  • You may have wasted a lot of time trying to memorize the names and types of cranial nerves but with little success. (allhealthpost.com)
  • Trigeminal nerve (CN V) Cranial nerve V covers most of the face. (theneurolight.com)
  • The main function of the trigeminal nerve is to transmit vital signals to the facial muscles and convey sensations such as pain, temperature, and touch from the face, mouth and the meninges. (allhealthpost.com)
  • The fifth cranial nerve is called the trigeminal nerve . (aapos.org)
  • There are twelve cranial nerves, but not all are included in the VASRD. (theneurolight.com)
  • Everyone has twelve cranial (brain) nerves. (nih.gov)
  • Abducent also goes by the name of external ocular motor cranial nerve. (allhealthpost.com)
  • Isolated third, fourth, and sixth cranial nerve palsies from presumed microvascular versus other causes: a prospective study. (nih.gov)
  • Third , fourth , and sixth cranial nerve palsies can limit eye movements and produce strabismus (misalignment of the eyes) and diplopia (double vision). (aapos.org)
  • Please refer to the specific FAQ sections on third , fourth , and sixth nerve palsies for specific information and recommendations (located on this website). (aapos.org)
  • These nerves control the movement of the eye muscles, focusing of the eyes, and constricting of the pupils. (coursehero.com)
  • They enter the skull at its base and travel with the vagus nerves to innervate two large neck muscles. (coursehero.com)
  • The cranial nerve innervation of some ventral neck muscles and the trapezius muscle to the scapula can be understood only in the context of their phylogenetic history beginning as the gill arch levator muscles of fish ( Romer & Parsons, 1986 ). (veteriankey.com)
  • It is a motor nerve and is responsible for carrying the signals to the extraocular muscles in the eyes which makes the lateral movements of the eyes possible. (allhealthpost.com)
  • The hypoglossal nerve functions like a motor nerve and is responsible for functions such as swallowing, speech, and muscles. (allhealthpost.com)
  • There are 12 pairs of cranial nerves and some are involved in special senses (sight, smell, hearing, taste, feeling) while others control muscles and glands. (aapos.org)
  • Cranial neuropathies can cause eye problems or problems with the muscles of the face. (nih.gov)
  • EMG shows how your muscles respond to your nerves. (nih.gov)
  • Cranial nerves I and II are the only two which do not exit the brain at the level of the brainstem. (theneurolight.com)
  • There are 12 paired cranial nerves that arise from the brainstem. (theneurolight.com)
  • The cranial nerves consist of 12 nerves that arise from the brainstem. (nursesyoumoveme.com)
  • It is one of the longest cranial nerves, originating in the brainstem and branching out throughout the face. (patheoushealth.com)
  • The vagus nerve has its origin in the medulla oblongata and exits the skull via the jugular foramen. (nih.gov)
  • These nerves come from the medulla oblongata and leave the skull to innervate the throat area. (coursehero.com)
  • These nerves arise from the medulla oblongata and travel to areas of the body below the head and neck region. (coursehero.com)
  • These nerves come from the medulla oblongata and innervate the tongue. (coursehero.com)
  • The glossopharyngeal nerve has its origin in the medulla oblongata and exits the skull via the jugular foramen, which is where the tympanic nerve branches off to give parasympathetic innervation to the parotid gland. (statpearls.com)
  • The Glossopharyngeal, Vagus and hypoglossal nerves are attached to the medulla oblongata. (nursesyoumoveme.com)
  • The pons is vital to the central and peripheral nervous system - a major reason for this being its connections to several cranial nerves including the trigeminal, abducens, facial, and vestibulocochlear nerves. (theneurolight.com)
  • V111 .Vestibulocochlear nerve is for Equilibrium and hearing. (nursesyoumoveme.com)
  • Vestibulocochlear nerve arise from the inner ear and travels to the pons. (nursesyoumoveme.com)
  • Cranial nerves pathways I-XII BY Prof. Dr. Abdul Waheed Ansari Chairperson & Prof. Anatomy, RAKCODS/RAKCOMS. (theneurolight.com)
  • The experts at UC San Diego Health diagnose and treat conditions involving the 12 pairs of cranial nerves that originate from the brain. (ucsd.edu)
  • The fibers originate in the inferior salivary nucleus then travel with the tympanic nerve through the foramen ovale, and synapse at the otic ganglion. (statpearls.com)
  • Cranial nerves are pathways that originate from the brain. (theneurolight.com)
  • [ 9 ] In neurofibromatosis 2 (NF2), the eighth cranial nerve is affected, and the classic finding is a bilateral vestibular schwannoma. (medscape.com)
  • Because of the wide distribution of the nerve throughout the body, there are several clinical correlations of the vagus nerve. (nih.gov)
  • the spinal accessory nerve (CN XI) joins the vagus nerve just distal to the inferior ganglion. (nih.gov)
  • The vagus nerve continues by traveling inferiorly within the carotid sheath where it is located posterior and lateral to the internal and common carotid arteries, and medial to the internal jugular vein. (nih.gov)
  • it makes its descent into the thoracic cavity by traveling to the right of the trachea, and posterior to the hilum on the right, moving medially to form the esophageal plexus with the left vagus nerve. (nih.gov)
  • it then descends posteriorly to the phrenic nerve and posterior to the left lung, then travels medially towards to the esophagus forming the esophageal plexus with the right vagus nerve. (nih.gov)
  • There are four branches of the vagus nerve within the neck: pharyngeal branches, superior laryngeal nerve, recurrent laryngeal nerve, and the superior cardiac nerve. (nih.gov)
  • The right recurrent laryngeal nerve's fibers branch from the vagus nerve near the right subclavian artery, traveling superiorly to enter the larynx between the cricopharyngeus muscle and the esophagus. (nih.gov)
  • The vagus nerve is a visceral motor in control of the heart, abdominal organs, and lungs. (legalinfo.com)
  • Its branches consist of tympanic, tonsillar, stylopharyngeal, carotid sinus nerve, branches to the tongue, lingual branches, and a communicating branch to cranial nerve X (vagus nerve). (statpearls.com)
  • [3] Additionally, one study showed that there was a small subset of the population that showed intracranial connections between the glossopharyngeal nerve and the vagus nerve, [4] which merits consideration during surgery of the posterior fossa to avoid transection of these nerve fibers. (statpearls.com)
  • Schwannomas are slow-growing, benign, encapsulated neoplasms that arise from the nerve sheath and consist of Schwann cells in a collagenous matrix. (medscape.com)
  • Schwannomas can arise from all cranial nerves except the optic. (medscape.com)
  • These nerves also arise from the pons and then innervate the face. (coursehero.com)
  • These nerves are unique in that they arise from the spinal cord, not the brain stem. (coursehero.com)
  • A cranial nerve is any of the 12 paired nerves that arise from the lower surface of the brain, with one of each pair on each side passing through openings in the skull to the periphery of the body. (legalinfo.com)
  • The cranial nerves are nerves that arise directly from the brain. (theneurolight.com)
  • Trigeminal,Abducens and Facial nerves arise in pons. (nursesyoumoveme.com)
  • Cranial nerves exit the inferior region of the cranium through foramina in the cranial bones and are numbered I-XII. (coursehero.com)
  • Cranial nerves are the 12 nerves of the peripheral nervous system that emerge from the foramina and fissures of the cranium. (theneurolight.com)
  • Cranial Nerve-VI Palsy as the Main Clinical Manifestation of Neurosarcoidosis. (nih.gov)
  • The diagnostic dilemma of neuro-imaging in acute isolated sixth nerve palsy. (nih.gov)
  • For exampe if the inflammation affects the facial nerve or the surrounding tissues, patients can develop facial nerve palsy. (picmonic.com)
  • HSV type 1 reactivation can sometimes result in facial nerve palsy. (picmonic.com)
  • Bell's palsy is the most common cause of facial nerve palsy. (picmonic.com)
  • The disease can involve multiple systems including the peripheral nervous system and can cause unilateral/bilateral facial nerve palsy, this usually occurs in the early disseminated stage. (picmonic.com)
  • Corticosteroids can be used for the treatment of facial nerve palsy if it is caused by inflammation and/or swelling of the nerve or its surrounding soft tissues. (picmonic.com)
  • A palsy is a lack of function of a nerve. (aapos.org)
  • A cranial nerve palsy may cause a partial weakness or complete paralysis of the areas served by the affected nerve. (aapos.org)
  • A cranial nerve palsy can occur due to a variety of causes. (aapos.org)
  • Patients with a cranial nerve palsy may adopt an abnormal head position to compensate for their strabismus and reduce diplopia. (aapos.org)
  • In addition to limited eye movements, a third nerve palsy can also cause ptosis (droopy eyelid) or mydriasis (an abnormally dilated pupil). (aapos.org)
  • 3. Diagnostic relevance of transcranial magnetic and electric stimulation of the facial nerve in the management of facial palsy. (nih.gov)
  • 4. The management of peripheral facial nerve palsy: "paresis" versus "paralysis" and sources of ambiguity in study designs. (nih.gov)
  • 16. Peripheral facial nerve palsy related to HIV infection: relationship with the immunological status and the HIV staging in Central Africa. (nih.gov)
  • 20. Recurrent attacks of facial nerve palsy as the presenting sign of leukemic relapse. (nih.gov)
  • vestibular schwannomas are the most common cranial nerve (CN) schwannomas, followed by trigeminal and facial schwannomas and then glossopharyngeal, vagus, and spinal accessory nerve schwannomas. (medscape.com)
  • It also goes by the names vestibular and auditory nerves. (allhealthpost.com)
  • the dorsal motor nucleus of X, superior ganglion of X, and the inferior ganglion of X. The nerve fibers from the nucleus ambiguous are efferent, special visceral (ESV) fibers which help to mediate swallowing and phonation. (nih.gov)
  • Vagus which is the tenth and most important Cranial nerve has the longest and varied area of supply, right from oral cavity to heart and other organs. (theneurolight.com)
  • Cranial Nerves The cranial nerves are composed of twelve pairs of nerves that emanate from the nervous tissue of the brain. (theneurolight.com)
  • There are 12 pairs of nerves in the Central Nerve System, a left and a right nerve for each nerve type. (nf2is.org)
  • Before the advent of magnetic resonance imaging (MRI), imaging of the cranial nerves was difficult, and mass lesions arising from these nerves was often indirectly detected only by looking at bony changes in the skull base foramen or by using invasive techniques such as cisternography and angiography. (medscape.com)
  • Cranial nerves emerge from or enter the skull, as opposed to the spinal nerves which emerge from the vertebral column. (theneurolight.com)
  • In fish and amphibians there are only 10 pairs of cranial nerves, because the succeeding nerves are not enclosed by the skull and hence are considered as spinal nerves. (veteriankey.com)
  • The first spinal nerve of fish is large and serves gill and opercular structures that are destined to be incorporated into the skull. (veteriankey.com)
  • Not until reptiles evolved did the development of the skull envelop the first and second spinal nerves, making them cranial nerves XI (accessory nerve) and XII (hypoglossal nerve). (veteriankey.com)
  • The 12 cranial nerves of the dog emerge from or enter the brain through foramina of the skull to innervate structures of the head and body. (veteriankey.com)
  • Cranial nerves are the 12 nerve pairs that emerge directly from the holes at the bottom of the skull without passing through the skull. (allhealthpost.com)
  • The glossopharyngeal nerve is the 9th cranial nerve (CN IX). (statpearls.com)
  • Special visceral efferent fibers (branchial motor) are the main motor fibers of the glossopharyngeal nerve and supply motor innervation to the stylopharyngeus muscle. (statpearls.com)
  • The glossopharyngeal nerve derives from the third pharyngeal arch and is the only structure formed by the third pharyngeal arch, although the third pharyngeal pouch is the embryologic origin of part of the thymus. (statpearls.com)
  • As stated above, the glossopharyngeal nerve provides motor innervation to the stylopharyngeus muscle, which is responsible for elevating the pharynx and larynx. (statpearls.com)
  • Some anatomic variations exist on the course of the glossopharyngeal nerve. (statpearls.com)
  • The glossopharyngeal nerve is the 9 th of the twelve pairs of cranial nerves. (theneurolight.com)
  • Glossopharyngeal Nerve. (theneurolight.com)
  • cranial nucleus nerves ambiguus emerge sve. (theneurolight.com)
  • These nerves take the visual stimuli from the photoreceptors and transfer them to the human brain and emerge in the area known as diencephalon. (allhealthpost.com)
  • These nerves extend from the pons to the face and assist with the sensations of touch, temperature, and pain. (coursehero.com)
  • These nerves come from the pons and help with eye movement. (coursehero.com)
  • Aspects of vision, like peripheral vision, are under the control of the optic cranial nerve (II). (theneurolight.com)
  • The peripheral nervous system is made up of all the nerves that travel from the spinal cord to the rest of the body. (theneurolight.com)
  • This type of nerve damage is less common than peripheral or autonomic neuropathy. (nih.gov)
  • The nerve travels widely throughout the body affecting several organ systems and regions of the body, such as the tongue, pharynx, heart, and gastrointestinal system. (nih.gov)
  • They can also occur in spinal nerve roots, flexor surfaces of the upper and lower extremities, and the tongue. (medscape.com)
  • Damage to the facial nerve can result in the loss of the taste sensation on one half of the tongue and the inability to produce tears. (legalinfo.com)
  • Lastly, there is the hypoglossal nerve, a somatic motor that controls the movement of the tongue. (legalinfo.com)
  • Hypoglossal nerve: This is the movement of tongue, swallowing and speech. (nursesyoumoveme.com)
  • V. Trigeminal = This is the largest cranial nerves and it is divided into three branches, which consist of the ophthalmic, maxillary and mandibular nerves. (nursesyoumoveme.com)
  • This is a mixed nerve consisting of several different nerve fibers and is responsible for functions such as creating facial movements and sending stimuli to the lacrimal and salivary glands. (allhealthpost.com)
  • A short quiz covering cranial nerves and structures innervated by them. (geekymedics.com)
  • All other nerves in the body run from the spinal cord. (legalinfo.com)
  • This nerve does not contain Schwann cells. (nf2is.org)
  • These are also referred to as common ocular motor nerve and are responsible for controlling the movement of the eyes and modulating the size of the pupils in different light conditions. (allhealthpost.com)
  • Detailed neurological evaluation, including electromyogram/nerve conduction and blink reflex study. (nih.gov)
  • The autonomic nervous system (most commonly understood as the fight/flight and freeze system) has 12 cranial nerves, 5 of which are responsible/necessary for social engagement (a feeling of safety, restoration, resiliency, and a desire to connect with others and not isolate). (wordpress.com)
  • Characteristics of the cranial nerves, which are numbered from anterior to posterior as they attach to the brain, are summarized in Table 1. (theneurolight.com)
  • Granulomas can then impinge on almost any organ or nerve, causing dysfunction. (picmonic.com)
  • Unlike other nerves that travel throughout the entire body, the cranial nerves are located only in the head and neck regions. (coursehero.com)
  • The 12 pairs of cranial nerves each serve different areas of the head and neck regions. (coursehero.com)
  • X1 .Accessory nerve = Movements of the neck and shoulder. (nursesyoumoveme.com)
  • These nerves are located in the lower part of the brain and also extend over to other areas outside the brain such as the neck and thorax. (allhealthpost.com)
  • Cranial nerve disorders can cause symptoms that are challenging to live with on a day-to-day basis. (ucsd.edu)
  • Different cranial nerves evoke different symptoms when they are injured. (legalinfo.com)
  • Although less than 10 percent of people with diabetes feel symptoms of carpal tunnel syndrome, about 25 percent of people with diabetes have some nerve compression at the wrist. (nih.gov)
  • Symptoms depend on which nerve is affected. (nih.gov)
  • Entrapments-focal neuropathies that involve trapped nerves-cause symptoms that begin gradually and get worse over time. (nih.gov)
  • Focal neuropathies that do not involve trapped nerves cause symptoms that begin suddenly and improve after several weeks or months. (nih.gov)
  • Doctors diagnose focal neuropathies by asking about your symptoms and performing tests, such as nerve conduction studies and electromyography (EMG) . (nih.gov)
  • Cranial nerve I is located closest to the frontal lobe, while cranial nerve XII is found closest to the brain stem. (coursehero.com)
  • It is often complicated with neuropathies, including the facial nerve neuropathy due to the microvascular ischemia and imbalance between nerve fiber damage and repair. (picmonic.com)
  • This nerve is also susceptible to paralysis, which can result in devastating social, financial, and mental consequences for the victim. (legalinfo.com)
  • 15. Delayed presentation of traumatic facial nerve (CN VII) paralysis. (nih.gov)
  • Entrapments occur when nerves become compressed or trapped in areas where nerves pass through narrow passages between bones and tissues. (nih.gov)
  • There are 12 pairs of cranial nerves, and their names and functions can be tough to memorize. (theneurolight.com)
  • Trochlear nerves have both the somatic and motor functions. (allhealthpost.com)
  • These nerves help in marinating the orientation and balance in space through the auditory functions. (allhealthpost.com)
  • It is primarily used for the treatment of herpes simplex virus infections, but it can also be used for the treatment of facial nerve lesions if the etiology is viral. (picmonic.com)

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