Disorders of one or more of the twelve cranial nerves. With the exception of the optic and olfactory nerves, this includes disorders of the brain stem nuclei from which the cranial nerves originate or terminate.
Twelve pairs of nerves that carry general afferent, visceral afferent, special afferent, somatic efferent, and autonomic efferent fibers.
Conditions which produce injury or dysfunction of the second cranial or optic nerve, which is generally considered a component of the central nervous system. Damage to optic nerve fibers may occur at or near their origin in the retina, at the optic disk, or in the nerve, optic chiasm, optic tract, or lateral geniculate nuclei. Clinical manifestations may include decreased visual acuity and contrast sensitivity, impaired color vision, and an afferent pupillary defect.
Diseases of the first cranial (olfactory) nerve, which usually feature anosmia or other alterations in the sense of smell and taste. Anosmia may be associated with NEOPLASMS; CENTRAL NERVOUS SYSTEM INFECTIONS; CRANIOCEREBRAL TRAUMA; inherited conditions; toxins; METABOLIC DISEASES; tobacco abuse; and other conditions. (Adams et al., Principles of Neurology, 6th ed, pp229-31)
Diseases of the tenth cranial nerve, including brain stem lesions involving its nuclei (solitary, ambiguus, and dorsal motor), nerve fascicles, and intracranial and extracranial course. Clinical manifestations may include dysphagia, vocal cord weakness, and alterations of parasympathetic tone in the thorax and abdomen.
Dysfunction of one or more cranial nerves causally related to a traumatic injury. Penetrating and nonpenetrating CRANIOCEREBRAL TRAUMA; NECK INJURIES; and trauma to the facial region are conditions associated with cranial nerve injuries.
Diseases of the sixth cranial (abducens) nerve or its nucleus in the pons. The nerve may be injured along its course in the pons, intracranially as it travels along the base of the brain, in the cavernous sinus, or at the level of superior orbital fissure or orbit. Dysfunction of the nerve causes lateral rectus muscle weakness, resulting in horizontal diplopia that is maximal when the affected eye is abducted and ESOTROPIA. Common conditions associated with nerve injury include INTRACRANIAL HYPERTENSION; CRANIOCEREBRAL TRAUMA; ISCHEMIA; and INFRATENTORIAL NEOPLASMS.
Pathological processes of the VESTIBULOCOCHLEAR NERVE, including the branches of COCHLEAR NERVE and VESTIBULAR NERVE. Common examples are VESTIBULAR NEURITIS, cochlear neuritis, and ACOUSTIC NEUROMA. Clinical signs are varying degree of HEARING LOSS; VERTIGO; and TINNITUS.
Diseases of the twelfth cranial (hypoglossal) nerve or nuclei. The nuclei and fascicles of the nerve are located in the medulla, and the nerve exits the skull via the hypoglossal foramen and innervates the muscles of the tongue. Lower brain stem diseases, including ischemia and MOTOR NEURON DISEASES may affect the nuclei or nerve fascicles. The nerve may also be injured by diseases of the posterior fossa or skull base. Clinical manifestations include unilateral weakness of tongue musculature and lingual dysarthria, with deviation of the tongue towards the side of weakness upon attempted protrusion.
Diseases of the ninth cranial (glossopharyngeal) nerve or its nuclei in the medulla. The nerve may be injured by diseases affecting the lower brain stem, floor of the posterior fossa, jugular foramen, or the nerve's extracranial course. Clinical manifestations include loss of sensation from the pharynx, decreased salivation, and syncope. Glossopharyngeal neuralgia refers to a condition that features recurrent unilateral sharp pain in the tongue, angle of the jaw, external auditory meatus and throat that may be associated with SYNCOPE. Episodes may be triggered by cough, sneeze, swallowing, or pressure on the tragus of the ear. (Adams et al., Principles of Neurology, 6th ed, p1390)
Diseases of the trigeminal nerve or its nuclei, which are located in the pons and medulla. The nerve is composed of three divisions: ophthalmic, maxillary, and mandibular, which provide sensory innervation to structures of the face, sinuses, and portions of the cranial vault. The mandibular nerve also innervates muscles of mastication. Clinical features include loss of facial and intra-oral sensation and weakness of jaw closure. Common conditions affecting the nerve include brain stem ischemia, INFRATENTORIAL NEOPLASMS, and TRIGEMINAL NEURALGIA.
Benign and malignant neoplasms that arise from one or more of the twelve cranial nerves.
Diseases of the oculomotor nerve or nucleus that result in weakness or paralysis of the superior rectus, inferior rectus, medial rectus, inferior oblique, or levator palpebrae muscles, or impaired parasympathetic innervation to the pupil. With a complete oculomotor palsy, the eyelid will be paralyzed, the eye will be in an abducted and inferior position, and the pupil will be markedly dilated. Commonly associated conditions include neoplasms, CRANIOCEREBRAL TRAUMA, ischemia (especially in association with DIABETES MELLITUS), and aneurysmal compression. (From Adams et al., Principles of Neurology, 6th ed, p270)
Diseases of the facial nerve or nuclei. Pontine disorders may affect the facial nuclei or nerve fascicle. The nerve may be involved intracranially, along its course through the petrous portion of the temporal bone, or along its extracranial course. Clinical manifestations include facial muscle weakness, loss of taste from the anterior tongue, hyperacusis, and decreased lacrimation.
Filarial infection of the eyes transmitted from person to person by bites of Onchocerca volvulus-infected black flies. The microfilariae of Onchocerca are thus deposited beneath the skin. They migrate through various tissues including the eye. Those persons infected have impaired vision and up to 20% are blind. The incidence of eye lesions has been reported to be as high as 30% in Central America and parts of Africa.
Diseases of the eleventh cranial (spinal accessory) nerve. This nerve originates from motor neurons in the lower medulla (accessory portion of nerve) and upper spinal cord (spinal portion of nerve). The two components of the nerve join and exit the skull via the jugular foramen, innervating the sternocleidomastoid and trapezius muscles, which become weak or paralyzed if the nerve is injured. The nerve is commonly involved in MOTOR NEURON DISEASE, and may be injured by trauma to the posterior triangle of the neck.
Diseases of the fourth cranial (trochlear) nerve or its nucleus in the midbrain. The nerve crosses as it exits the midbrain dorsally and may be injured along its course through the intracranial space, cavernous sinus, superior orbital fissure, or orbit. Clinical manifestations include weakness of the superior oblique muscle which causes vertical DIPLOPIA that is maximal when the affected eye is adducted and directed inferiorly. Head tilt may be seen as a compensatory mechanism for diplopia and rotation of the visual axis. Common etiologies include CRANIOCEREBRAL TRAUMA and INFRATENTORIAL NEOPLASMS.
The 7th cranial nerve. The facial nerve has two parts, the larger motor root which may be called the facial nerve proper, and the smaller intermediate or sensory root. Together they provide efferent innervation to the muscles of facial expression and to the lacrimal and SALIVARY GLANDS, and convey afferent information for TASTE from the anterior two-thirds of the TONGUE and for TOUCH from the EXTERNAL EAR.
Inflammation of the optic nerve. Commonly associated conditions include autoimmune disorders such as MULTIPLE SCLEROSIS, infections, and granulomatous diseases. Clinical features include retro-orbital pain that is aggravated by eye movement, loss of color vision, and contrast sensitivity that may progress to severe visual loss, an afferent pupillary defect (Marcus-Gunn pupil), and in some instances optic disc hyperemia and swelling. Inflammation may occur in the portion of the nerve within the globe (neuropapillitis or anterior optic neuritis) or the portion behind the globe (retrobulbar neuritis or posterior optic neuritis).
A nerve which originates in the lumbar and sacral spinal cord (L4 to S3) and supplies motor and sensory innervation to the lower extremity. The sciatic nerve, which is the main continuation of the sacral plexus, is the largest nerve in the body. It has two major branches, the TIBIAL NERVE and the PERONEAL NERVE.
The 9th cranial nerve. The glossopharyngeal nerve is a mixed motor and sensory nerve; it conveys somatic and autonomic efferents as well as general, special, and visceral afferents. Among the connections are motor fibers to the stylopharyngeus muscle, parasympathetic fibers to the parotid glands, general and taste afferents from the posterior third of the tongue, the nasopharynx, and the palate, and afferents from baroreceptors and CHEMORECEPTOR CELLS of the carotid sinus.
The 3d cranial nerve. The oculomotor nerve sends motor fibers to the levator muscles of the eyelid and to the superior rectus, inferior rectus, and inferior oblique muscles of the eye. It also sends parasympathetic efferents (via the ciliary ganglion) to the muscles controlling pupillary constriction and accommodation. The motor fibers originate in the oculomotor nuclei of the midbrain.
The nerves outside of the brain and spinal cord, including the autonomic, cranial, and spinal nerves. Peripheral nerves contain non-neuronal cells and connective tissue as well as axons. The connective tissue layers include, from the outside to the inside, the epineurium, the perineurium, and the endoneurium.
Traumatic injuries to the HYPOGLOSSAL NERVE.
The 5th and largest cranial nerve. The trigeminal nerve is a mixed motor and sensory nerve. The larger sensory part forms the ophthalmic, mandibular, and maxillary nerves which carry afferents sensitive to external or internal stimuli from the skin, muscles, and joints of the face and mouth and from the teeth. Most of these fibers originate from cells of the TRIGEMINAL GANGLION and project to the TRIGEMINAL NUCLEUS of the brain stem. The smaller motor part arises from the brain stem trigeminal motor nucleus and innervates the muscles of mastication.
The 6th cranial nerve which originates in the ABDUCENS NUCLEUS of the PONS and sends motor fibers to the lateral rectus muscles of the EYE. Damage to the nerve or its nucleus disrupts horizontal eye movement control.
The 8th cranial nerve. The vestibulocochlear nerve has a cochlear part (COCHLEAR NERVE) which is concerned with hearing and a vestibular part (VESTIBULAR NERVE) which mediates the sense of balance and head position. The fibers of the cochlear nerve originate from neurons of the SPIRAL GANGLION and project to the cochlear nuclei (COCHLEAR NUCLEUS). The fibers of the vestibular nerve arise from neurons of Scarpa's ganglion and project to the VESTIBULAR NUCLEI.
The 2nd cranial nerve which conveys visual information from the RETINA to the brain. The nerve carries the axons of the RETINAL GANGLION CELLS which sort at the OPTIC CHIASM and continue via the OPTIC TRACTS to the brain. The largest projection is to the lateral geniculate nuclei; other targets include the SUPERIOR COLLICULI and the SUPRACHIASMATIC NUCLEI. Though known as the second cranial nerve, it is considered part of the CENTRAL NERVOUS SYSTEM.
Slender processes of NEURONS, including the AXONS and their glial envelopes (MYELIN SHEATH). Nerve fibers conduct nerve impulses to and from the CENTRAL NERVOUS SYSTEM.
A syndrome of congenital facial paralysis, frequently associated with abducens palsy and other congenital abnormalities including lingual palsy, clubfeet, brachial disorders, cognitive deficits, and pectoral muscle defects. Pathologic findings are variable and include brain stem nuclear aplasia, facial nerve aplasia, and facial muscle aplasia, consistent with a multifactorial etiology. (Adams et al., Principles of Neurology, 6th ed, p1020)
Mechanical compression of nerves or nerve roots from internal or external causes. These may result in a conduction block to nerve impulses (due to MYELIN SHEATH dysfunction) or axonal loss. The nerve and nerve sheath injuries may be caused by ISCHEMIA; INFLAMMATION; or a direct mechanical effect.
The 11th cranial nerve which originates from NEURONS in the MEDULLA and in the CERVICAL SPINAL CORD. It has a cranial root, which joins the VAGUS NERVE (10th cranial) and sends motor fibers to the muscles of the LARYNX, and a spinal root, which sends motor fibers to the TRAPEZIUS and the sternocleidomastoid muscles.
Traumatic injuries to the LARYNGEAL NERVE.
Paralysis of one or more of the ocular muscles due to disorders of the eye muscles, neuromuscular junction, supporting soft tissue, tendons, or innervation to the muscles.
Severe or complete loss of facial muscle motor function. This condition may result from central or peripheral lesions. Damage to CNS motor pathways from the cerebral cortex to the facial nuclei in the pons leads to facial weakness that generally spares the forehead muscles. FACIAL NERVE DISEASES generally results in generalized hemifacial weakness. NEUROMUSCULAR JUNCTION DISEASES and MUSCULAR DISEASES may also cause facial paralysis or paresis.
Renewal or physiological repair of damaged nerve tissue.
The 4th cranial nerve. The trochlear nerve carries the motor innervation of the superior oblique muscles of the eye.
A syndrome characterized by recurrent episodes of excruciating pain lasting several seconds or longer in the sensory distribution of the TRIGEMINAL NERVE. Pain may be initiated by stimulation of trigger points on the face, lips, or gums or by movement of facial muscles or chewing. Associated conditions include MULTIPLE SCLEROSIS, vascular anomalies, ANEURYSMS, and neoplasms. (Adams et al., Principles of Neurology, 6th ed, p187)
A general term most often used to describe severe or complete loss of muscle strength due to motor system disease from the level of the cerebral cortex to the muscle fiber. This term may also occasionally refer to a loss of sensory function. (From Adams et al., Principles of Neurology, 6th ed, p45)
Junction between the cerebellum and the pons.
Neoplasms of the base of the skull specifically, differentiated from neoplasms of unspecified sites or bones of the skull (SKULL NEOPLASMS).
Traumatic injuries to the facial nerve. This may result in FACIAL PARALYSIS, decreased lacrimation and salivation, and loss of taste sensation in the anterior tongue. The nerve may regenerate and reform its original pattern of innervation, or regenerate aberrantly, resulting in inappropriate lacrimation in response to gustatory stimuli (e.g., "crocodile tears") and other syndromes.
Traumatic injuries to the TROCHLEAR NERVE.
The 12th cranial nerve. The hypoglossal nerve originates in the hypoglossal nucleus of the medulla and supplies motor innervation to all of the muscles of the tongue except the palatoglossus (which is supplied by the vagus). This nerve also contains proprioceptive afferents from the tongue muscles.
A paraganglioma involving the glomus jugulare, a microscopic collection of chemoreceptor tissue in the adventitia of the bulb of the jugular vein. It may cause paralysis of the vocal cords, attacks of dizziness, blackouts, and nystagmus. It is not resectable but radiation therapy is effective. It regresses slowly, but permanent control is regularly achieved. (From Dorland, 27th ed; Stedman, 25th ed; DeVita Jr et al., Cancer: Principles & Practice of Oncology, 3d ed, pp1603-4)
Interruption of NEURAL CONDUCTION in peripheral nerves or nerve trunks by the injection of a local anesthetic agent (e.g., LIDOCAINE; PHENOL; BOTULINUM TOXINS) to manage or treat pain.
Branch-like terminations of NERVE FIBERS, sensory or motor NEURONS. Endings of sensory neurons are the beginnings of afferent pathway to the CENTRAL NERVOUS SYSTEM. Endings of motor neurons are the terminals of axons at the muscle cells. Nerve endings which release neurotransmitters are called PRESYNAPTIC TERMINALS.
The dense rock-like part of temporal bone that contains the INNER EAR. Petrous bone is located at the base of the skull. Sometimes it is combined with the MASTOID PROCESS and called petromastoid part of temporal bone.
A branch of the tibial nerve which supplies sensory innervation to parts of the lower leg and foot.
An irregularly shaped venous space in the dura mater at either side of the sphenoid bone.
A major nerve of the upper extremity. In humans, the fibers of the median nerve originate in the lower cervical and upper thoracic spinal cord (usually C6 to T1), travel via the brachial plexus, and supply sensory and motor innervation to parts of the forearm and hand.
Treatment of muscles and nerves under pressure as a result of crush injuries.
Injuries to the PERIPHERAL NERVES.
The medial terminal branch of the sciatic nerve. The tibial nerve fibers originate in lumbar and sacral spinal segments (L4 to S2). They supply motor and sensory innervation to parts of the calf and foot.
The inferior region of the skull consisting of an internal (cerebral), and an external (basilar) surface.
A major nerve of the upper extremity. In humans, the fibers of the ulnar nerve originate in the lower cervical and upper thoracic spinal cord (usually C7 to T1), travel via the medial cord of the brachial plexus, and supply sensory and motor innervation to parts of the hand and forearm.
A visual symptom in which a single object is perceived by the visual cortex as two objects rather than one. Disorders associated with this condition include REFRACTIVE ERRORS; STRABISMUS; OCULOMOTOR NERVE DISEASES; TROCHLEAR NERVE DISEASES; ABDUCENS NERVE DISEASES; and diseases of the BRAIN STEM and OCCIPITAL LOBE.
The infratentorial compartment that contains the CEREBELLUM and BRAIN STEM. It is formed by the posterior third of the superior surface of the body of the sphenoid (SPHENOID BONE), by the occipital, the petrous, and mastoid portions of the TEMPORAL BONE, and the posterior inferior angle of the PARIETAL BONE.
A nerve originating in the lumbar spinal cord (usually L2 to L4) and traveling through the lumbar plexus to provide motor innervation to extensors of the thigh and sensory innervation to parts of the thigh, lower leg, and foot, and to the hip and knee joints.
Non-invasive method of demonstrating internal anatomy based on the principle that atomic nuclei in a strong magnetic field absorb pulses of radiofrequency energy and emit them as radiowaves which can be reconstructed into computerized images. The concept includes proton spin tomographic techniques.
The 31 paired peripheral nerves formed by the union of the dorsal and ventral spinal roots from each spinal cord segment. The spinal nerve plexuses and the spinal roots are also included.

Hypoglossal nerve injury as a complication of anterior surgery to the upper cervical spine. (1/228)

Injury to the hypoglossal nerve is a recognised complication after soft tissue surgery in the upper part of the anterior aspect of the neck, e.g. branchial cyst or carotid body tumour excision. However, this complication has been rarely reported following surgery of the upper cervical spine. We report the case of a 35-year-old woman with tuberculosis of C2-3. She underwent corpectomy and fusion from C2 to C5 using iliac crest bone graft, through a left anterior oblique incision. She developed hypoglossal nerve palsy in the immediate postoperative period, with dysphagia and dysarthria. It was thought to be due to traction neurapraxia with possible spontaneous recovery. At 18 months' follow-up, she had a solid fusion and tuberculosis was controlled. The hypoglossal palsy persisted, although with minimal functional disability. The only other reported case of hypoglossal lesion after anterior cervical spine surgery in the literature also failed to recover. It is concluded that hypoglossal nerve palsy following anterior cervical spine surgery is unlikely to recover spontaneously and it should be carefully identified.  (+info)

MR of CNS sarcoidosis: correlation of imaging features to clinical symptoms and response to treatment. (2/228)

BACKGROUND AND PURPOSE: Sarcoidosis is an idiopathic systemic granulomatous disease, recognized in a patient when clinical and radiologic findings are confirmed by histopathologic analysis. The objective was to identify a relationship between MR imaging and clinical findings in CNS sarcoidosis. METHODS: The clinical charts of 461 patients with biopsy-proved sarcoidosis were reviewed retrospectively. Criteria for including patients in the study included those with symptoms referable to the CNS, excluding those with another explanation for their symptoms, those with headaches or other subjective complaints without accompanying objective findings, and those with peripheral neuropathy other than cranial nerve involvement or myopathy without CNS manifestations. Thirty-four of 38 patients whose conditions met the criteria for CNS sarcoidosis underwent a total of 82 MR examinations. The positive imaging findings were divided into categories as follows: pachymeningeal, leptomeningeal, nonenhancing brain parenchymal, enhancing brain parenchymal, cranial nerve, and spinal cord and nerve root involvement. Treatment response, clinical symptomatology, and any available histopathologic studies were analyzed with respect to imaging manifestations in each of the categories. RESULTS: Eighty-two percent of the patients with sarcoidosis with neurologic symptoms referable to the CNS had findings revealed by MR imaging. However, eight (40%) of 20 cranial nerve deficits seen at clinical examination of 13 patients were not seen at contrast-enhanced MR imaging, and 50% of the patients with symptoms referable to the pituitary axis had no abnormal findings on routine contrast-enhanced MR images. In contradistinction, 44% of 18 cranial nerves in nine patients with MR evidence of involvement had no symptoms referable to the involved cranial nerve. Clinical and radiologic deterioration occurred more commonly with leptomeningeal and enhancing brain parenchymal lesions. CONCLUSION: MR imaging can be used to confirm clinical suspicion and to show subclinical disease and the response of pathologic lesions to treatment.  (+info)

Clinical and MRI study of brain stem and cerebellar involvement in Japanese patients with multiple sclerosis. (3/228)

OBJECTIVES: To investigate the clinical and MRI features of brain stem and cerebellar lesions in Japanese patients with multiple sclerosis. METHODS: A retrospective study of 66 consecutive Japanese patients with multiple sclerosis (42 women and 24 men) was done by reviewing the medical records and MRI films. Forty nine patients were diagnosed as having clinically definite multiple sclerosis and 17 patients as having clinically probable multiple sclerosis according to Poser's criteria. Prevalence rates of each brain stem and cerebellar manifestation and frequency and distribution of MRI lesions in these patients were studied. RESULTS: Forty three patients (65%) had one or more infratentorial manifestations. Cranial nerves were clinically involved in 28 patients (42%), and most of the lesions were identified by MRI. Among them, manifestations of facial, trigeminal, and abducens nerves were relatively common. Cerebellar ataxia was found in 20 patients (30%). The MRI study showed that the lesions responsible for ataxia in these patients were mainly found in the cerebellar peduncles, but cerebellar hemispheric lesions were detected in only four patients (6.4%). CONCLUSION: The low frequency (6.4%) of the cerebellar MRI lesions in these patients is in sharp contrast with the figures reported for white patients with multiple sclerosis (50%-90%). Racial and genetic differences may have an influence on the susceptibility of each part of the CNS to demyelination in multiple sclerosis.  (+info)

CNS involvement in children with newly diagnosed non-Hodgkin's lymphoma. (4/228)

PURPOSE: To determine the frequency of CNS involvement at diagnosis of non-Hodgkin's lymphoma (NHL), to characterize its pattern of presentation, and to determine its prognostic significance. PATIENTS AND METHODS: We reviewed the records of 445 children (1975 through 1995) diagnosed with NHL (small noncleaved cell NHL/B-cell acute lymphoblastic leukemia [SNCC NHL/B-ALL], 201 patients; lymphoblastic, 113; large cell, 119; other, 12). Tumor burden was estimated by serum lactate dehydrogenase (LDH) measurement and reclassification of disease stage irrespective of CNS involvement (modified stage). RESULTS: Thirty-six of 445 children with newly diagnosed NHL had CNS involvement (lymphoma cells in the CSF [n = 23], cranial nerve palsy [n = 9], both features [n = 4]), representing 13%, 7%, and 1% of small noncleaved cell lymphoma, lymphoblastic lymphoma, and large-cell cases, respectively. By univariate analysis, CNS disease at diagnosis did not significantly impact event-free survival (P =. 095), whereas stage and LDH did; however, children with CNS disease at diagnosis were at 2.0 times greater risk of death than those without CNS disease at diagnosis. In a multivariate analysis, CNS disease was not significantly associated with either overall or event-free survival, whereas both serum LDH and stage influenced both overall and event-free survival. Among cases of SNCC NHL/B-ALL, CNS disease was significantly associated with event-free and overall survival (univariate analysis); however, in multivariate analysis, only LDH had independent prognostic significance. Elevated serum LDH or higher modified stage were associated with a trend toward poorer overall survival among children with CNS disease. CONCLUSION: A greater tumor burden at diagnosis adversely influences the treatment outcome of children with NHL and CNS disease at diagnosis, suggesting a need for ongoing improvement in both systemic and CNS-directed therapy.  (+info)

Corneal structure and sensitivity in type 1 diabetes mellitus. (5/228)

PURPOSE: Corneal wound healing is impaired in diabetic cornea. The purpose of this study was to examine patients with type 1 diabetes mellitus for changes in corneal morphology and to correlate corneal sensitivity, subbasal nerve morphology, and degree of polyneuropathy with each other. METHODS: Forty-four eyes of 23 patients with diabetes and nine control eyes were included. Corneal sensitivity was tested with a Cochet-Bonnet esthesiometer (Luneau, Paris, France), and corneal morphology and epithelial and corneal thickness were determined by in vivo confocal microscopy. The density of subbasal nerves was evaluated by calculating the number of long subbasal nerve fiber bundles per confocal microscopic field. The degree of polyneuropathy was evaluated using the clinical part of the Michigan Neuropathy Screening Instrument (MNSI) classification, and retinopathy was evaluated using fundus photographs. RESULTS: A reduction of long nerve fiber bundles per image was noted to have occurred already in patients with mild to moderate neuropathy, but corneal mechanical sensitivity was reduced only in patients with severe neuropathy. Compared with control subjects the corneal thickness was increased in patients with diabetes without neuropathy. The epithelium of patients with diabetes with severe neuropathy was significantly thinner than that of patients with diabetes without neuropathy. CONCLUSIONS: Confocal microscopy appears to allow early detection of beginning neuropathy, because decreases in nerve fiber bundle counts precede impairment of corneal sensitivity. Apparently, the cornea becomes thicker in a relatively early stage of diabetes but does not further change with the degree of neuropathy. A reduction in neurotrophic stimuli in severe neuropathy may induce a thin epithelium that may lead to recurrent erosions.  (+info)

An unusual case of multiple cranial nerve palsies in Wegener's granulomatosis. (6/228)

We describe an unusual case of Wegener's granulomatosis, which initially caused fulminant palsies affecting cranial nerves II, V, VI, VII, and VIII during a brief episode of the disease. The patient was successfully treated with immunosuppressive therapy. Wegener's granulomatosis should be suspected when multiple cranial nerves are initially affected.  (+info)

Corneal morphology and sensitivity in lattice dystrophy type II (familial amyloidosis, Finnish type). (7/228)

PURPOSE: To describe the corneal abnormalities and to measure different modalities of corneal sensitivity in corneal lattice dystrophy type II (familial amyloidosis, Finnish type, also known as gelsolin-related amyloidosis and originally as Meretoja syndrome). METHODS: Twenty eyes of 20 patients were examined by in vivo confocal microscopy and noncontact gas esthesiometry. RESULTS: Pleomorphism of, and dense deposits between or posterior to, the basal epithelial cells were frequently observed, as well as a reduction of long nerve fiber bundles in the subbasal nerve plexus. The anterior stroma was altered in most cases, with fibrosis and abnormal extracellular matrix. In 15 corneas, thick anterior and midstromal filaments, corresponding to lattice lines, and in 11 corneas, thin undulated structures were observed. The average mechanical sensitivity threshold of 12 subjects was increased, and in the remaining 8 subjects there was no response, even to the highest intensity of stimuli used. Three patients did not respond to CO(2), 11 to heat, and 2 to cold, but those patients who responded had normal thresholds. Patients with more long nerve fiber bundles per confocal microscopic image had better mechanical and cold sensitivity than patients with fewer nerve fiber bundles. CONCLUSIONS: Lattice lines seem to be related to amyloid material and not to corneal nerves. However, the subbasal nerve density appears reduced, which results mainly in a decrease in mechanical and, to a lesser extent, thermal sensitivity. The location of stromal filaments and undulated structures changes with increasing age.  (+info)

Parapharyngeal second branchial cyst manifesting as cranial nerve palsies: MR findings. (8/228)

SUMMARY: We report the MR findings of parapharyngeal branchial cleft cyst manifesting as multiple, lower cranial nerve palsies in a 35-year-old woman. On MR images, a well-marginated cystic mass was detected in the right parapharyngeal space, with displacement of both the right internal carotid artery and the right internal jugular vein on the posterolateral side. The cyst contained a whitish fluid that was slightly hyperintense on T1-weighted images and slightly hypointense to CSF on T2-weighted images. No enhancement on contrast-enhanced T1-weighted images was present. The right side of the tongue showed high signal intensity on T2-weighted images, suggesting denervation.  (+info)

Some common examples of cranial nerve diseases include:

1. Bell's palsy: A condition that affects the facial nerve, causing weakness or paralysis of one side of the face.
2. Multiple sclerosis: An autoimmune disease that damages the protective covering of nerve fibers, leading to communication problems between the brain and the rest of the body.
3. Trigeminal neuralgia: A condition that affects the trigeminal nerve, causing facial pain and numbness.
4. Meningitis: An inflammation of the meninges, the protective covering of the brain and spinal cord, which can damage the cranial nerves.
5. Acoustic neuroma: A type of non-cancerous tumor that grows on the nerve that connects the inner ear to the brain.
6. Cranial polyneuropathy: A condition where multiple cranial nerves are damaged, leading to a range of symptoms including muscle weakness, numbness, and pain.
7. Tumors: Both benign and malignant tumors can affect the cranial nerves, causing a variety of symptoms depending on their location and size.
8. Trauma: Head injuries or trauma can damage the cranial nerves, leading to a range of symptoms.
9. Infections: Bacterial or viral infections such as meningitis or encephalitis can damage the cranial nerves, leading to a range of symptoms.
10. Genetic disorders: Certain genetic disorders such as Charcot-Marie-Tooth disease can affect the cranial nerves, leading to a range of symptoms.

It's important to note that this is not an exhaustive list and there may be other causes of cranial nerve damage. If you are experiencing any symptoms that you think may be related to cranial nerve damage, it's important to seek medical attention as soon as possible for proper diagnosis and treatment.

The olfactory nerve is responsible for transmitting sensory information from the nose to the brain, allowing us to perceive and distinguish different smells. Olfactory nerve diseases refer to any disorders or damage that affects this nerve, leading to impaired smell function or other symptoms.

Types of Olfactory Nerve Diseases:

1. Olfactory neurodegeneration: This condition is characterized by progressive loss of olfactory sensory neurons and can lead to complete loss of smell.
2. Olfactory hallucinations: This rare disorder causes individuals to perceive strange or distorted smells, often accompanied by other neurological symptoms.
3. Phantosmia (olfactory hallucinations): This condition is characterized by the perception of foul or unusual odors that are not actually present.
4. Parosmia (distortion of smell): This disorder is characterized by the distortion or alteration of the perception of smells, often accompanied by other neurological symptoms.
5. Anosmia (loss of smell): This condition is characterized by the complete loss of the ability to perceive smells.
6. Hyposmia (decreased sense of smell): This disorder is characterized by a decreased ability to perceive smells, which can be caused by various factors such as nasal polyps, allergies, or neurological conditions.
7. Hyperosmia (increased sensitivity to smell): This condition is characterized by an increased sensitivity to smells, which can be caused by various factors such as hormonal changes, allergies, or neurological conditions.

Causes of Olfactory Nerve Diseases:

1. Viral infections: Some viral infections such as the common cold and influenza can damage the olfactory nerve and lead to olfactory dysfunction.
2. Bacterial infections: Certain bacterial infections such as meningitis and sinusitis can also damage the olfactory nerve and cause olfactory dysfunction.
3. Trauma: A head injury or trauma to the nose and sinuses can damage the olfactory nerve and lead to olfactory dysfunction.
4. Allergies: Allergic reactions can cause inflammation and irritation of the nasal passages, leading to olfactory dysfunction.
5. Nasal polyps: Growths in the nasal passages, such as nasal polyps, can obstruct the olfactory nerve and lead to olfactory dysfunction.
6. Neurodegenerative diseases: Certain neurodegenerative diseases such as Alzheimer's disease, Parkinson's disease, and Huntington's disease can damage the olfactory nerve and lead to olfactory dysfunction.
7. Tumors: Tumors in the nasal passages or brain can cause damage to the olfactory nerve and lead to olfactory dysfunction.
8. Vitamin deficiencies: Deficiencies in vitamins such as vitamin B12 and zinc can affect the functioning of the olfactory nerve and lead to olfactory dysfunction.
9. Aging: As people age, the sense of smell can decline due to changes in the olfactory system and brain.
10. Genetics: Some people may be born with a genetic predisposition to olfactory dysfunction, which can be caused by mutations in genes that are involved in the functioning of the olfactory system.

It's important to note that olfactory dysfunction can have a significant impact on quality of life, as it can affect an individual's ability to taste and enjoy food, detect danger signals such as smoke or gas leaks, and even affect their social interactions and relationships. If you suspect that you or someone you know is experiencing olfactory dysfunction, it is important to seek medical attention to determine the underlying cause and develop a treatment plan.

1. Vagus nerve paralysis: A condition in which the vagus nerve is damaged or degenerated, leading to weakness or paralysis of the muscles involved in swallowing and breathing.
2. Vagus nerve neuritis: Inflammation of the vagus nerve, which can cause symptoms such as hoarseness, dysphagia, and pain in the throat.
3. Vagus nerve tumors: Abnormal growths on the vagus nerve that can cause a variety of symptoms, including difficulty swallowing, voice changes, and seizures.
4. Vagus nerve trauma: Damage to the vagus nerve due to injury or surgery, which can result in long-term consequences such as dysphagia and vocal cord paralysis.
5. Vagus nerve syndromes: A group of disorders that affect the vagus nerve and its connections with other organs, such as the heart and lungs. These syndromes can cause a range of symptoms, including seizures, difficulty breathing, and abnormal heart rhythms.

These are some examples of Vagus Nerve Diseases that can affect the quality of life of an individual. It is important to be aware of these conditions and seek medical attention if symptoms persist or worsen over time.

Types of Cranial Nerve Injuries:

1. Traumatic brain injury (TBI): TBI can cause damage to the cranial nerves, leading to a range of symptoms such as double vision, facial weakness or paralysis, difficulty with swallowing, and cognitive impairment.
2. Stroke: A stroke can cause damage to the cranial nerves, leading to symptoms such as a drooping eyelid, facial weakness or paralysis, and difficulty with swallowing.
3. Brain tumors: Tumors in the brain can compress or damage the cranial nerves, causing a range of symptoms such as double vision, facial weakness or paralysis, and cognitive impairment.
4. Cerebral vasospasm: This is a condition where the blood vessels in the brain constrict, reducing blood flow and oxygen supply to the brain, which can cause damage to the cranial nerves.
5. Infections such as meningitis or encephalitis: These infections can cause inflammation of the membranes surrounding the brain and spinal cord, leading to damage to the cranial nerves.
6. Neurodegenerative diseases such as Parkinson's disease, multiple sclerosis, and amyotrophic lateral sclerosis (ALS): These conditions can cause progressive damage to the cranial nerves leading to a range of symptoms such as tremors, weakness, and difficulty with movement and balance.

Symptoms of Cranial Nerve Injuries:

1. Double vision or loss of vision
2. Facial weakness or paralysis
3. Difficulty with swallowing
4. Slurred speech
5. Weakness or paralysis of the limbs on one side of the body
6. Difficulty with balance and coordination
7. Numbness or tingling in the face, arms, or legs
8. Seizures
9. Vision problems such as blurred vision, loss of peripheral vision, or loss of color vision
10. Cognitive impairment such as difficulty with concentration, memory loss, or difficulty with problem-solving.

Diagnosis of Cranial Nerve Injuries:

1. Physical examination and medical history: A doctor will perform a physical examination to check for signs of cranial nerve damage such as weakness or paralysis of the facial muscles, difficulty with swallowing, or abnormal reflexes.
2. Imaging tests such as CT or MRI scans: These tests can help doctors identify any structural problems in the brain or spinal cord that may be causing cranial nerve damage.
3. Electromyography (EMG) and nerve conduction studies (NCS): These tests can help doctors determine the extent of nerve damage by measuring the electrical activity of muscles and nerves.
4. Lumbar puncture: This test involves inserting a needle into the spinal canal to collect cerebrospinal fluid for laboratory testing.
5. Blood tests: These can help doctors rule out other conditions that may be causing symptoms such as infections or autoimmune disorders.

Treatment of Cranial Nerve Injuries:

1. Conservative management: Mild cases of cranial nerve injuries may not require surgical intervention and can be treated with conservative measures such as physical therapy, pain management, and monitoring.
2. Surgery: In more severe cases, surgery may be necessary to relieve compression on the nerves or repair any structural damage.
3. Rehabilitation: After surgery or conservative treatment, rehabilitation is crucial to regain lost function and prevent further complications. This may include physical therapy, occupational therapy, and speech therapy.

Prognosis of Cranial Nerve Injuries:

The prognosis for cranial nerve injuries depends on the severity and location of the injury, as well as the promptness and effectiveness of treatment. In general, the sooner treatment is received, the better the outcome. Some people may experience a full recovery, while others may have persistent symptoms or long-term deficits.

Complications of Cranial Nerve Injuries:

1. Permanent nerve damage: In some cases, cranial nerve injuries can result in permanent nerve damage, leading to chronic symptoms such as weakness, numbness, or paralysis.
2. Seizures: Cranial nerve injuries can increase the risk of seizures, particularly if they involve the seizure-regulating nerves.
3. Infection: Any injury that penetrates the skull can increase the risk of infection, which can be life-threatening if left untreated.
4. Hydrocephalus: This is a condition in which cerebrospinal fluid accumulates in the brain, leading to increased intracranial pressure and potentially life-threatening complications.
5. Cerebral edema: This is swelling of the brain tissue due to injury or inflammation, which can lead to increased intracranial pressure and potentially life-threatening complications.
6. Brain herniation: This is a condition in which the brain is pushed out of its normal position in the skull, leading to potentially life-threatening complications.
7. Vision loss: Cranial nerve injuries can cause vision loss or blindness, particularly if they involve the optic nerves.
8. Facial paralysis: Cranial nerve injuries can cause facial paralysis or weakness, which can be temporary or permanent.
9. Hearing loss: Cranial nerve injuries can cause hearing loss or deafness, particularly if they involve the auditory nerves.
10. Cognitive and behavioral changes: Depending on the location and severity of the injury, cranial nerve injuries can lead to cognitive and behavioral changes, such as difficulty with concentration, memory problems, or personality changes.

In summary, cranial nerve injuries can have a significant impact on an individual's quality of life, and it is important to seek medical attention immediately if symptoms persist or worsen over time.

Some common abducens nerve diseases include:

1. Abducens paresis or palsy: This is a weakness or paralysis of the abducens nerve that can cause difficulty moving the eyeball outward or away from the nose.
2. Brown syndrome: This is a condition where the nerve is compressed or damaged, leading to difficulty moving the eye laterally.
3. Congenital abducens palsy: This is a condition present at birth that affects the development of the abducens nerve and can result in limited or absent movement of one or both eyes.
4. Trauma to the abducens nerve: This can occur due to head injuries, facial trauma, or other forms of injury that damage the nerve.
5. Tumors or cysts: Growths in the orbit or near the abducens nerve can compress or damage the nerve and cause abducens nerve diseases.
6. Inflammatory conditions: Conditions such as Graves' disease, multiple sclerosis, or sarcoidosis can inflame the nerve and cause abducens nerve diseases.
7. Stroke or cerebral vasculature disorders: These conditions can damage the nerve due to reduced blood flow or bleeding in the brain.

Symptoms of abducens nerve diseases may include double vision, difficulty moving one or both eyes, and difficulty focusing. Diagnosis is typically made through a combination of physical examination, imaging studies such as MRI or CT scans, and electrophysiological tests such as electromyography. Treatment options vary depending on the underlying cause of the disease and may include glasses or contact lenses for double vision, prism lenses to align the eyes, or surgery to correct any anatomical abnormalities. In some cases, medications such as steroids or immunosuppressants may be prescribed to reduce inflammation and promote healing.

Some examples of vestibulocochlear nerve diseases include:

1. Meniere's disease: A disorder of the inner ear that causes vertigo, tinnitus, hearing loss, and a feeling of fullness in the affected ear.
2. Acoustic neuroma: A benign tumor that grows on the vestibular nerve and can cause hearing loss, tinnitus, and balance difficulties.
3. Otosclerosis: A condition in which there is abnormal bone growth in the middle ear that can cause hearing loss and tinnitus.
4. Presbycusis: Age-related hearing loss that affects the inner ear and causes gradual hearing loss over time.
5. Sudden sensorineural hearing loss: A condition where an individual experiences sudden and significant hearing loss in one or both ears with no known cause.
6. Meningitis: Inflammation of the membranes that cover the brain and spinal cord, which can affect the vestibulocochlear nerve and cause hearing loss and balance difficulties.
7. Certain medications: Certain antibiotics, chemotherapy drugs, and aspirin at high doses can damage the inner ear and cause temporary or permanent hearing loss.
8. Trauma to the head or ear: A head injury or a sudden blow to the ear can cause damage to the vestibulocochlear nerve and result in hearing loss or balance difficulties.

These are some of the common examples of vestibulocochlear nerve diseases, but there are other rarer conditions that can also affect the vestibulocochlear nerve. A comprehensive evaluation by an otolaryngologist (ENT specialist) and a hearing specialist is necessary for proper diagnosis and treatment.

The hypoglossal nerve is a cranial nerve that controls the movement of the tongue and other muscles in the throat. Hypoglossal nerve diseases refer to conditions that affect the functioning of this nerve, leading to symptoms such as difficulty swallowing, weakness or paralysis of the tongue, and speech difficulties.

Some examples of hypoglossal nerve diseases include:

1. Hypoglossal neuritis: This is an inflammation of the hypoglossal nerve, which can be caused by viral infections, head injuries, or other conditions.
2. Hypoglossal nerve palsy: This is a condition where the hypoglossal nerve is damaged or compressed, leading to weakness or paralysis of the tongue and other muscles in the throat.
3. Congenital hypoglossal nerve defects: These are birth defects that affect the development of the hypoglossal nerve, leading to a range of symptoms including difficulty swallowing and speech difficulties.
4. Trauma to the hypoglossal nerve: This can occur due to injury or trauma to the neck or head, leading to weakness or paralysis of the tongue and other muscles in the throat.
5. Tumors or cysts affecting the hypoglossal nerve: These can cause compression or damage to the nerve, leading to symptoms such as difficulty swallowing, speech difficulties, and weakness or paralysis of the tongue.

Hypoglossal nerve diseases can be diagnosed through a range of tests, including electromyography (EMG), nerve conduction studies (NCS), and imaging studies such as MRI or CT scans. Treatment depends on the underlying cause of the condition and may include physical therapy, medication, or surgery.

Types of Glossopharyngeal Nerve Diseases:

1. Glossopharyngeal Neuralgia: This is a condition characterized by recurring episodes of sudden, severe pain in the tongue, throat, and ears. The pain can be triggered by coughing, swallowing, or other movements.
2. Glossopharyngeal Nerve Palsy: This is a condition where the glossopharyngeal nerve is damaged, leading to weakness or paralysis of the tongue and other muscles in the throat and mouth.
3. Glossopharyngeal Fibrillation: This is a condition characterized by rapid, involuntary contractions of the muscles in the throat, which can cause difficulty swallowing and other symptoms.

Causes of Glossopharyngeal Nerve Diseases:

1. Trauma to the head or neck
2. Viral infections such as herpes zoster (shingles) or Lyme disease
3. Bacterial infections such as meningitis or abscesses
4. Tumors or cysts in the throat or brain
5. Inflammatory conditions such as rheumatoid arthritis or sarcoidosis
6. Genetic disorders such as Charcot-Marie-Tooth disease or other inherited neurological conditions

Symptoms of Glossopharyngeal Nerve Diseases:

1. Pain in the tongue, throat, and ears
2. Weakness or paralysis of the tongue and other muscles in the throat and mouth
3. Difficulty swallowing (dysphagia)
4. Hoarseness or other changes in voice quality
5. Loss of taste sensation
6. Ear pain or hearing loss
7. Fatigue, weakness, or numbness in the face and neck
8. Involuntary movements of the tongue, lips, or jaw (tics)
9. Difficulty articulating speech
10. Coughing or choking on food or liquids.

Diagnosis of Glossopharyngeal Nerve Diseases:

1. Medical history and physical examination
2. Imaging studies such as CT or MRI scans
3. Electromyography (EMG) to test the function of muscles in the throat and face
4. Nerve conduction studies (NCS) to evaluate the function of nerves
5. Biopsy of tissue samples from the throat or neck to rule out other conditions.

Treatment for Glossopharyngeal Nerve Diseases:

1. Medications such as antiviral, antibiotic, or anti-inflammatory drugs
2. Surgery to remove tumors or treat nerve damage
3. Physical therapy to improve swallowing and speaking difficulties
4. Speech therapy to improve communication skills
5. Lifestyle changes such as avoiding irritants, maintaining good oral hygiene, and eating a balanced diet.

Prognosis for Glossopharyngeal Nerve Diseases:

The prognosis for glossopharyngeal nerve diseases varies depending on the underlying cause and severity of the condition. In general, with proper treatment and management, many patients can experience significant improvement in their symptoms and quality of life. However, some patients may experience persistent or recurrent symptoms, and in rare cases, the condition can be life-threatening. Early diagnosis and treatment are essential to achieve the best possible outcomes.

The trigeminal nerve is a cranial nerve that carries sensation from the face and head to the brain. Trigeminal nerve diseases are conditions that affect this nerve, leading to a range of symptoms such as pain, numbness, weakness, and difficulty with facial movements.

Types of Trigeminal Nerve Diseases:

1. Trigeminal Neuralgia: This is a chronic pain disorder that affects the trigeminal nerve, causing episodes of sudden, intense pain in the face, particularly around the eye and mouth.
2. Multiple Sclerosis (MS): MS is an autoimmune disease that can damage the trigeminal nerve, leading to pain, numbness, and weakness in the face.
3. Trigeminal Neuropathy: This is a condition where the trigeminal nerve is damaged due to injury, inflammation, or infection, leading to pain, numbness, and tingling in the face.
4. Bell's Palsy: This is a condition that affects the facial nerve, leading to weakness or paralysis of the muscles on one side of the face.
5. Herpes Zoster Oticus: This is a viral infection that affects the nerves in the ear and face, causing pain, numbness, and tingling in the face.

Symptoms of Trigeminal Nerve Diseases:

1. Pain: The most common symptom of trigeminal nerve diseases is pain, which can range from mild to severe and can be described as aching, burning, or electric-like.
2. Numbness or tingling: Patients may experience numbness or tingling sensations in the face, particularly around the eye and mouth.
3. Weakness: Some patients may experience weakness or paralysis of the muscles in the face, which can affect their ability to smile, talk, or eat.
4. Difficulty with facial movements: Trigeminal nerve diseases can cause difficulty with facial movements such as closing the eyes, smiling, or whistling.
5. Drooping eyelid or eyebrow: Some patients may experience drooping of the eyelid or eyebrow, which can be a sign of a more severe condition.
6. Eye problems: Trigeminal nerve diseases can cause eye problems such as double vision, blurred vision, or loss of vision in one eye.
7. Headaches: Patients may experience headaches or migraines due to the pressure or inflammation on the nerve.
8. Fatigue: Trigeminal nerve diseases can cause fatigue and exhaustion, particularly if the patient is experiencing persistent pain or discomfort.

Diagnosis of Trigeminal Nerve Diseases:

1. Medical history and physical examination: A thorough medical history and physical examination are essential to diagnose trigeminal nerve diseases.
2. Imaging studies: Imaging studies such as MRI or CT scans may be ordered to rule out other conditions and visualize the nerve.
3. Nerve conduction studies: Nerve conduction studies can help identify the specific location and extent of the nerve damage.
4. Blood tests: Blood tests may be ordered to check for signs of inflammation or infection.
5. Biopsy: A biopsy may be performed to obtain a tissue sample for further examination.

Treatment of Trigeminal Nerve Diseases:

1. Pain management: Pain management is the primary goal of treatment, and it can be achieved through medications, injections, or nerve blocks.
2. Anticonvulsants: Anticonvulsants may be prescribed to manage pain and prevent seizures.
3. Anti-inflammatory medications: Anti-inflammatory medications may be used to reduce inflammation and swelling.
4. Muscle relaxants: Muscle relaxants may be prescribed to relieve muscle spasms and tension.
5. Physical therapy: Physical therapy can help improve range of motion, strength, and function.
6. Surgery: In some cases, surgery may be necessary to relieve compression or damage to the nerve.

Prevention of Trigeminal Nerve Diseases:

1. Early diagnosis and treatment: Early diagnosis and treatment can help prevent progression of the disease and reduce symptoms.
2. Avoiding triggers: Avoiding triggers such as allergens, infections, or trauma can help prevent the onset of trigeminal nerve diseases.
3. Good oral hygiene: Maintaining good oral hygiene can help prevent infections that can lead to trigeminal nerve damage.
4. Avoiding repetitive motions: Avoiding repetitive motions such as frequent clenching or grinding of the teeth can help prevent nerve damage.
5. Proper body mechanics: Maintaining proper body mechanics and posture can help reduce strain on the nerve.
6. Regular check-ups: Regular check-ups with a healthcare professional can help detect any underlying conditions and prevent complications.

The symptoms of cranial nerve neoplasms depend on the location and size of the tumor, but may include:

* Headaches
* Pain in the face or head
* Numbness or weakness in the arms or legs
* Difficulty with vision, hearing, or balance
* Double vision
* Nausea and vomiting

Cranial nerve neoplasms can be diagnosed through a variety of tests, including:

* Imaging studies such as MRI or CT scans
* Biopsy, where a sample of tissue is removed for examination under a microscope
* Neurological examination to assess vision, hearing, balance, and other functions.

Treatment options for cranial nerve neoplasms depend on the location, size, and type of tumor, as well as the patient's overall health. Treatment may include:

* Surgery to remove the tumor
* Radiation therapy to kill any remaining cancer cells
* Chemotherapy to kill cancer cells
* Targeted therapy to attack specific molecules on the surface of cancer cells
* Observation, with regular monitoring and check-ups to see if the tumor is growing or changing.

It's important to note that cranial nerve neoplasms are relatively rare, and the prognosis and treatment options can vary depending on the specific type of tumor and the patient's overall health. A healthcare professional should be consulted for an accurate diagnosis and appropriate treatment plan.

Damage or dysfunction of the oculomotor nerve can result in a range of symptoms, including double vision (diplopia), drooping eyelids (ptosis), difficulty moving the eyes (ophthalmoplegia), and vision loss. The specific symptoms depend on the location and extent of the damage to the nerve.

Some common causes of oculomotor nerve diseases include:

1. Trauma or injury to the head or neck
2. Tumors or cysts in the brain or skull
3. Inflammatory conditions such as multiple sclerosis or sarcoidosis
4. Vasculitis or other blood vessel disorders
5. Certain medications, such as anticonvulsants or chemotherapy drugs
6. Nutritional deficiencies, such as vitamin B12 deficiency
7. Infections, such as meningitis or encephalitis
8. Genetic disorders, such as hereditary oculopharyngeal dystrophy
9. Ischemic or hemorrhagic strokes
10. Neurodegenerative diseases, such as Parkinson's disease or amyotrophic lateral sclerosis (ALS).

The diagnosis of oculomotor nerve diseases typically involves a comprehensive eye exam, neurological evaluation, and imaging studies such as MRI or CT scans. Treatment depends on the underlying cause and may include medications, surgery, or other interventions to address the underlying condition and relieve symptoms. In some cases, surgical intervention may be necessary to repair or replace damaged portions of the nerve.

Some examples of Facial Nerve Diseases include:

* Bell's Palsy: A condition that causes weakness or paralysis of the facial muscles on one side of the face, often resulting in drooping or twitching of the eyelid and facial muscles.
* Facial Spasm: A condition characterized by involuntary contractions of the facial muscles, which can cause twitching or spasms.
* Progressive Bulbar Palsy (PBP): A rare disorder that affects the brain and spinal cord, leading to weakness and wasting of the muscles in the face, tongue, and throat.
* Parry-Romberg Syndrome: A rare condition characterized by progressive atrophy of the facial muscles on one side of the face, leading to a characteristic "smile" or "grimace."
* Moebius Syndrome: A rare neurological disorder that affects the nerves responsible for controlling eye movements and facial expressions.
* Trauma to the Facial Nerve: Damage to the facial nerve can result in weakness or paralysis of the facial muscles, depending on the severity of the injury.

These are just a few examples of Facial Nerve Diseases, and there are many other conditions that can affect the facial nerve and cause similar symptoms. A comprehensive diagnosis and evaluation by a healthcare professional is necessary to determine the specific underlying condition and develop an appropriate treatment plan.

The parasite migrates to various tissues throughout the body, including the skin, subcutaneous tissue, and eyes. In the eye, the parasite can cause inflammation and damage to the retina, optic nerve, and choroid, leading to visual impairment and blindness.

The most common form of ocular onchocerciasis is trachoma, which affects the conjunctiva and cornea. Trachoma is responsible for 2.8% of all global blindness and 9.6% of all infectious blindness.

Ocular onchocerciasis can be diagnosed through a combination of physical examination, imaging studies, and laboratory tests, such as PCR or ELISA. Treatment options include antiparasitic drugs, such as ivermectin, which is effective against the adult worms, and surgery to remove inflamed tissue.

Prevention of ocular onchocerciasis includes vector control measures, such as using insecticides to kill infected blackflies, and mass drug administration (MDA) programs to eliminate the parasite in endemic areas.

Examples of Accessory Nerve Diseases:

1. Accessory nerve paralysis: A condition where the accessory nerve is damaged or compressed, leading to weakness or paralysis of the muscles it supplies.
2. Accessory nerve neuritis: Inflammation of the accessory nerve, which can cause pain and weakness in the neck and arm.
3. Accessory nerve lesions: Damage to the accessory nerve, often caused by trauma or compression, can result in muscle weakness or paralysis.
4. Accessory nerve tumors: Tumors that grow on the accessory nerve can cause pain, weakness, and numbness or tingling in the neck and arm.
5. Accessory nerve cysts: Fluid-filled sacs that form on the accessory nerve can cause pain and weakness in the affected area.

Symptoms of Accessory Nerve Diseases:

* Weakness or paralysis of the muscles of the neck, shoulder, and arm
* Pain in the neck and arm
* Numbness or tingling sensations in the skin of the neck and arm
* Difficulty swallowing or speaking (in severe cases)

Diagnosis of Accessory Nerve Diseases:

* Physical examination to test muscle strength and reflexes
* Electromyography (EMG) to measure the electrical activity of muscles
* Imaging studies, such as X-rays or MRI, to rule out other conditions
* Nerve conduction studies to test the function of the accessory nerve

Treatment of Accessory Nerve Diseases:

* Physical therapy to improve muscle strength and range of motion
* Medications to manage pain and inflammation
* Surgery to relieve compression or repair damaged tissue
* Electrical stimulation therapy to promote nerve regeneration

It's important to note that the treatment for accessory nerve diseases can vary depending on the underlying cause of the condition. In some cases, surgery may be necessary to relieve compression or repair damaged tissue. In other cases, physical therapy and medication may be sufficient to manage symptoms and improve function.

In conclusion, the accessory nerve plays a crucial role in controlling the muscles of the neck and arm. Accessory nerve diseases can cause weakness, pain, and numbness or tingling sensations in the affected area. Early diagnosis and treatment are essential to manage symptoms and improve function.

Example sentences:

1. The patient was diagnosed with a trochlear nerve disease that caused her eyes to drift apart, making it difficult for her to read or focus on objects.
2. The doctor specialized in treating trochlear nerve diseases and had seen many patients with similar symptoms but different causes.
3. The surgery was successful in repairing the damage to the trochlear nerve and restoring the patient's vision.

Note: Trochlear nerve diseases can be caused by various factors, including trauma, tumors, stroke, multiple sclerosis, and more. They can also be inherited or congenital.

The symptoms of optic neuritis may include:

* Blurred vision or loss of vision
* Eye pain or pressure
* Sensitivity to light
* Dimness of colors
* Difficulty moving the eyes
* Numbness or weakness in the face

The cause of optic neuritis is not always known, but it is believed to be related to an abnormal immune response. In MS, optic neuritis is thought to be triggered by the immune system attacking the protective covering of nerve fibers in the central nervous system.

Treatment for optic neuritis depends on the underlying cause. In cases of MS, treatment with corticosteroids can help reduce inflammation and slow the progression of the disease. In other conditions, treatment may involve addressing the underlying cause, such as an infection or a tumor.

Prognosis for optic neuritis varies depending on the underlying cause. In MS, the condition can recur and lead to long-term vision loss if left untreated. However, with prompt treatment and management, many people with MS experience significant improvement in their vision.

The hypoglossal nerve is a cranial nerve that controls the movement of the tongue and is responsible for its protrusion, withdrawal, and lateral movement. Hypoglossal nerve injuries can occur due to various reasons such as trauma, surgery, or tumors. These injuries can result in symptoms such as tongue weakness or paralysis, difficulty speaking or swallowing, and loss of taste sensation on the tip of the tongue.

The severity of hypoglossal nerve injuries can vary from mild to severe, and the treatment options depend on the cause and extent of the injury. Mild cases may resolve on their own with time, while more severe cases may require surgical intervention or other treatments such as physical therapy or medications. In this article, we will discuss the causes, symptoms, diagnosis, and treatment options for hypoglossal nerve injuries in detail.

Causes of Hypoglossal Nerve Injuries:

Hypoglossal nerve injuries can occur due to various reasons such as:

Trauma: Traumatic injuries to the face or neck can cause damage to the hypoglossal nerve, resulting in tongue weakness or paralysis.

Surgery: Surgical procedures in the head and neck region can sometimes result in injury to the hypoglossal nerve.

Tumors: Tumors in the head and neck region can compress or injure the hypoglossal nerve, leading to tongue weakness or paralysis.

Infections: Infections such as meningitis or abscesses in the head and neck region can damage the hypoglossal nerve.

Neurodegenerative diseases: Certain neurodegenerative diseases such as amyotrophic lateral sclerosis (ALS) can affect the hypoglossal nerve, leading to tongue weakness or paralysis.

Symptoms of Hypoglossal Nerve Injuries:

The symptoms of hypoglossal nerve injuries can vary depending on the severity and location of the injury. Common symptoms include:

Tongue weakness or paralysis: Weakness or paralysis of the tongue can make it difficult to speak, eat, or swallow.

Drooling: Inability to control salivation due to tongue weakness or paralysis can lead to drooling.

Difficulty articulating words: Slurred speech or difficulty articulating words due to tongue weakness or paralysis.

Facial weakness or paralysis: Weakness or paralysis of the facial muscles can cause drooping or weakness in the face.

Difficulty swallowing: Difficulty swallowing due to tongue weakness or paralysis can lead to dysphagia.

Causes of Hypoglossal Nerve Injuries:

Hypoglossal nerve injuries can occur due to various reasons, including:

Trauma: Traumatic injuries to the face or neck can cause damage to the hypoglossal nerve, resulting in tongue weakness or paralysis.

Surgery: Surgical procedures in the head and neck region can sometimes cause nerve damage, leading to hypoglossal nerve injuries.

Neurological conditions: Certain neurological conditions such as stroke, multiple sclerosis, or tumors can cause hypoglossal nerve injuries.

Viral infections: Viral infections such as HIV or Lyme disease can cause inflammation of the nerves, including the hypoglossal nerve.

Treatment options for Hypoglossal Nerve Injuries:

Treatment options for hypoglossal nerve injuries depend on the underlying cause and severity of the injury. Some possible treatment options include:

Physical therapy: Physical therapy exercises can help improve tongue strength and mobility.

Medications: Medications such as antiviral drugs or steroids may be prescribed to manage symptoms.

Surgery: In some cases, surgery may be necessary to relieve compression or repair damaged nerve tissue.

Speech therapy: Speech therapy can help improve communication skills and address swallowing difficulties.

It's important to seek medical attention if you experience any symptoms of hypoglossal nerve injuries, as prompt treatment can help prevent long-term complications and improve outcomes.

The symptoms of Mobius syndrome are caused by damage to the sixth and seventh cranial nerves, which control facial movements and swallowing. The disorder is usually inherited as an autosomal dominant trait, meaning that a single copy of the mutated gene is enough to cause the condition. However, some cases may occur spontaneously due to a genetic mutation or other factors.

There is no cure for Mobius syndrome, but treatment can help manage the symptoms and improve quality of life. Physical therapy, occupational therapy, and speech therapy may be recommended to help improve muscle strength and coordination, as well as communication skills. In some cases, surgery may be necessary to correct physical deformities or relieve pressure on the brain and spinal cord.

The prognosis for Mobius syndrome varies depending on the severity of the condition and the presence of any other underlying health issues. Some individuals with Mobius syndrome may have a relatively mild form of the disorder, while others may experience more severe symptoms and functional limitations. With appropriate treatment and support, many people with Mobius syndrome are able to lead fulfilling lives and achieve their goals.

There are several types of nerve compression syndromes, including:

1. Carpal tunnel syndrome: Compression of the median nerve in the wrist, commonly caused by repetitive motion or injury.
2. Tarsal tunnel syndrome: Compression of the posterior tibial nerve in the ankle, similar to carpal tunnel syndrome but affecting the lower leg.
3. Cubital tunnel syndrome: Compression of the ulnar nerve at the elbow, often caused by repetitive leaning or bending.
4. Thoracic outlet syndrome: Compression of the nerves and blood vessels that pass through the thoracic outlet (the space between the neck and shoulder), often caused by poor posture or injury.
5. Peripheral neuropathy: A broader term for damage to the peripheral nerves, often caused by diabetes, vitamin deficiencies, or other systemic conditions.
6. Meralgia paresthetica: Compression of the lateral femoral cutaneous nerve in the thigh, commonly caused by direct trauma or compression from a tight waistband or clothing.
7. Morton's neuroma: Compression of the plantar digital nerves between the toes, often caused by poorly fitting shoes or repetitive stress on the feet.
8. Neuralgia: A general term for pain or numbness caused by damage or irritation to a nerve, often associated with chronic conditions such as shingles or postherpetic neuralgia.
9. Trigeminal neuralgia: A condition characterized by recurring episodes of sudden, extreme pain in the face, often caused by compression or irritation of the trigeminal nerve.
10. Neuropathic pain: Pain that occurs as a result of damage or dysfunction of the nervous system, often accompanied by other symptoms such as numbness, tingling, or weakness.

There are several types of ophthalmoplegia, including:

1. External ophthalmoplegia: This type affects the muscles that control lateral and vertical movements of the eyes.
2. Internal ophthalmoplegia: This type affects the muscles that control rotational movements of the eyes.
3. Superior oblique paresis: This type affects the superior oblique muscle, which controls downward and outward movements of the eye.
4. Inferior oblique paresis: This type affects the inferior oblique muscle, which controls upward and outward movements of the eye.

Symptoms of ophthalmoplegia may include difficulty moving the eyes, double vision, droopy eyelids, and blurred vision. Treatment options depend on the underlying cause of the condition and may include physical therapy, prism lenses, or surgery.

The main symptoms of facial paralysis are:

1. Weakness or numbness in the facial muscles
2. Drooping or sagging of one side of the face
3. Twitching or spasms in the facial muscles
4. Difficulty smiling, frowning, or expressing emotions
5. Difficulty closing the eye on the affected side
6. Dry mouth or difficulty swallowing
7. Pain or discomfort in the face or head.

The diagnosis of facial paralysis is based on a combination of clinical examination, imaging studies such as MRI or CT scans, and other tests to determine the underlying cause. Treatment options for facial paralysis depend on the underlying cause and may include medications, surgery, physical therapy, and other interventions to address any associated symptoms.

There are several types of facial paralysis, including:

1. Bell's palsy: A condition that causes weakness or paralysis of the muscles on one side of the face, usually due to nerve damage.
2. Facial spasm: A condition characterized by involuntary twitching or contractions of the facial muscles.
3. Hemifacial spasm: A condition that causes weakness or paralysis of half of the face due to nerve compression.
4. Trauma-related facial paralysis: Caused by injury or trauma to the face or head.
5. Tumor-related facial paralysis: Caused by a tumor that compresses or damages the nerves responsible for facial movement.
6. Stroke-related facial paralysis: Caused by a stroke that affects the nerves responsible for facial movement.
7. Neurodegenerative diseases such as Parkinson's disease, multiple sclerosis, and amyotrophic lateral sclerosis (ALS).
8. Infection-related facial paralysis: Caused by infections such as Lyme disease, meningitis, or encephalitis.
9. Post-viral facial paralysis: Caused by a viral infection that affects the nerves responsible for facial movement.

Treatment for facial paralysis depend on the underlying cause and may include medications, surgery, physical therapy, and other interventions to address any associated symptoms.

The symptoms of TN can vary in severity and frequency, and may include:

* Pain on one side of the face
* Episodes of sudden, intense pain that can be triggered by light touch or contact with the face
* Pain that is described as stabbing, shooting, or like an electric shock
* Spontaneous pain episodes without any apparent cause
* Pain that is worse with light sensation, such as from wind, cold, or touch
* Pain that is better with pressing or rubbing the affected area

The exact cause of TN is not known, but it is believed to be related to compression or irritation of the trigeminal nerve. The condition can be caused by a variety of factors, including:

* A blood vessel pressing on the nerve
* A tumor or cyst in the brain or face
* Multiple sclerosis or other conditions that damage the nerve
* Injury to the nerve
* Genetic mutations that affect the nerve

There is no cure for TN, but various treatments can help manage the symptoms. These may include:

* Medications such as anticonvulsants or pain relievers
* Nerve blocks or injections to reduce inflammation and relieve pain
* Surgery to decompress the nerve or remove a tumor or cyst
* Lifestyle modifications, such as avoiding triggers and using gentle, soothing touch

It is important for individuals with TN to work closely with their healthcare provider to find the most effective treatment plan for their specific needs. With proper management, many people with TN are able to experience significant relief from their symptoms and improve their quality of life.

1. Complete paralysis: When there is no movement or sensation in a particular area of the body.
2. Incomplete paralysis: When there is some movement or sensation in a particular area of the body.
3. Localized paralysis: When paralysis affects only a specific part of the body, such as a limb or a facial muscle.
4. Generalized paralysis: When paralysis affects multiple parts of the body.
5. Flaccid paralysis: When there is a loss of muscle tone and the affected limbs feel floppy.
6. Spastic paralysis: When there is an increase in muscle tone and the affected limbs feel stiff and rigid.
7. Paralysis due to nerve damage: This can be caused by injuries, diseases such as multiple sclerosis, or birth defects such as spina bifida.
8. Paralysis due to muscle damage: This can be caused by injuries, such as muscular dystrophy, or diseases such as muscular sarcopenia.
9. Paralysis due to brain damage: This can be caused by head injuries, stroke, or other conditions that affect the brain such as cerebral palsy.
10. Paralysis due to spinal cord injury: This can be caused by trauma, such as a car accident, or diseases such as polio.

Paralysis can have a significant impact on an individual's quality of life, affecting their ability to perform daily activities, work, and participate in social and recreational activities. Treatment options for paralysis depend on the underlying cause and may include physical therapy, medications, surgery, or assistive technologies such as wheelchairs or prosthetic devices.

Types of Skull Base Neoplasms:

1. Meningioma: A benign tumor that arises from the meninges, the protective membranes covering the brain and spinal cord.
2. Acoustic neuroma (vestibular schwannoma): A benign tumor that grows on the nerve that connects the inner ear to the brain.
3. Pineal parenchymal tumors: Tumors that occur in the pineal gland, a small endocrine gland located in the brain.
4. Craniopharyngiomas: Benign tumors that arise from the cells of the pituitary gland and the hypothalamus.
5. Chordomas: Malignant tumors that arise from the cells of the notochord, a structure that gives rise to the spinal cord.
6. Chondrosarcomas: Malignant tumors that arise from cartilage cells.
7. Osteosarcomas: Malignant tumors that arise from bone cells.
8. Melanotic neuroectodermal tumors: Rare tumors that are usually benign but can sometimes be malignant.

Causes and Symptoms of Skull Base Neoplasms:

The exact cause of skull base neoplasms is not always known, but they can be associated with genetic mutations or exposure to certain environmental factors. Some of the symptoms of skull base neoplasms include:

* Headaches
* Vision problems
* Hearing loss
* Balance and coordination difficulties
* Seizures
* Weakness or numbness in the face or limbs
* Endocrine dysfunction (in case of pituitary tumors)

Diagnosis of Skull Base Neoplasms:

The diagnosis of skull base neoplasms usually involves a combination of imaging studies such as CT or MRI scans, and tissue sampling through biopsy or surgery. The specific diagnostic tests will depend on the location and symptoms of the tumor.

Treatment of Skull Base Neoplasms:

The treatment of skull base neoplasms depends on the type, size, location, and aggressiveness of the tumor, as well as the patient's overall health. Some of the treatment options for skull base neoplasms include:

* Surgery: The primary treatment for most skull base neoplasms is surgical resection. The goal of surgery is to remove as much of the tumor as possible while preserving as much normal tissue as possible.
* Radiation therapy: Radiation therapy may be used before or after surgery to shrink the tumor and kill any remaining cancer cells.
* Chemotherapy: Chemotherapy may be used in combination with radiation therapy to treat skull base neoplasms that are aggressive or have spread to other parts of the body.
* Endoscopic surgery: Endoscopic surgery is a minimally invasive procedure that uses a thin, lighted tube with a camera on the end (endoscope) to remove the tumor through the nasal cavity or sinuses.
* Stereotactic radiosurgery: Stereotactic radiosurgery is a non-invasive procedure that uses highly focused radiation beams to destroy the tumor. It is typically used for small, well-defined tumors that are located in sensitive areas of the skull base.

Prognosis for Skull Base Neoplasms:

The prognosis for skull base neoplasms depends on the type and location of the tumor, as well as the patient's overall health. In general, the prognosis for patients with skull base neoplasms is good if the tumor is small, located in a accessible area, and has not spread to other parts of the body. However, the prognosis may be poorer for patients with larger or more aggressive tumors, or those that have spread to other parts of the body.

It's important to note that each patient is unique and the prognosis can vary depending on individual circumstances. It is best to consult a medical professional for specific information about the prognosis for your condition.

There are several types of facial nerve injuries, including:

1. Bell's palsy: This is a condition that affects the facial nerve and causes weakness or paralysis of the muscles on one side of the face. It is often temporary and resolves on its own within a few weeks.
2. Facial paralysis: This is a condition in which the facial nerve is damaged, leading to weakness or paralysis of the muscles of facial expression. It can be caused by trauma, tumors, or viral infections.
3. Ramsay Hunt syndrome: This is a rare condition that occurs when the facial nerve is affected by a virus, leading to symptoms such as facial paralysis and pain in the ear.
4. Traumatic facial nerve injury: This can occur as a result of trauma to the head or face, such as a car accident or a fall.
5. Tumor-related facial nerve injury: In some cases, tumors can grow on the facial nerve and cause damage.
6. Ischemic facial nerve injury: This occurs when there is a reduction in blood flow to the facial nerve, leading to damage to the nerve fibers.
7. Neurofibromatosis type 2: This is a rare genetic disorder that can cause tumors to grow on the facial nerve, leading to damage and weakness of the facial muscles.

Treatment for facial nerve injuries depends on the underlying cause and severity of the injury. In some cases, physical therapy may be recommended to help regain strength and control of the facial muscles. Surgery may also be necessary in some cases to repair damaged nerve fibers or remove tumors.

The trochlear nerve is a small nerve that originates in the brain and passes through the base of the skull, down through the orbit (eye socket), and into the eye. It provides sensation to the superior oblique muscle, which is responsible for rotating the eye downward and outward. Injuries to the trochlear nerve can cause a variety of symptoms, including double vision, drooping of the eyelid, and difficulty moving the eyes.

The most common causes of trochlear nerve injuries include:

1. Trauma to the head or eye
2. Inflammation of the orbit (orbital inflammation)
3. Tumors in the orbit or brain
4. Cranial or orbital fractures
5. Infections such as meningitis or encephalitis
6. Stroke or cerebral vasculature disorders
7. Neurodegenerative diseases such as Parkinson's disease, multiple sclerosis, or myasthenia gravis.

Symptoms of trochlear nerve injuries can include:

1. Double vision (diplopia)
2. Drooping of the eyelid (ptosis)
3. Difficulty moving the eyes (oculomotor dysfunction)
4. Pain or discomfort in the eye or orbit
5. Redness or swelling of the conjunctiva (the thin membrane covering the white part of the eye)
6. Difficulty closing the eyelid completely
7. Sensitivity to light (photophobia)
8. Blurred vision
9. Nausea and vomiting
10. Headache

Diagnosis of trochlear nerve injuries is typically made through a combination of physical examination, imaging studies such as CT or MRI scans, and specialized tests such as electromyography (EMG) or visual evoked potentials (VEP).

Treatment of trochlear nerve injuries depends on the underlying cause and severity of the injury. In some cases, surgery may be necessary to repair the nerve or relieve compression on the nerve. Physical therapy and exercises may also be recommended to help restore strength and range of motion in the affected eye. Medications such as anticholinergics or anti-inflammatory drugs may be prescribed to relieve symptoms such as dryness, redness, or pain. In cases where the injury is caused by a more serious condition such as a tumor or aneurysm, treatment of the underlying condition may be necessary before addressing the nerve injury.

Glomus jugulare tumors are thought to arise from abnormal growth of cells called glomus cells, which are found in the walls of blood vessels throughout the body. These cells play a role in regulating blood pressure by producing certain hormones and chemicals that help to constrict or dilate blood vessels.

Glomus jugulare tumors can cause a variety of symptoms depending on their size and location, including:

1. Swelling in the neck or face
2. Pain in the neck or face
3. Difficulty swallowing
4. Numbness or weakness in the face
5. Vision problems

If a glomus jugulare tumor is suspected, a doctor may perform several tests to confirm the diagnosis and determine the best course of treatment. These tests may include:

1. Ultrasound: This non-invasive imaging test uses sound waves to create pictures of the blood vessels in the neck.
2. Computed tomography (CT) scan: This test uses X-rays and computer technology to create detailed images of the neck and head.
3. Magnetic resonance imaging (MRI): This test uses a strong magnetic field and radio waves to create detailed images of the blood vessels in the neck.
4. Biopsy: In this test, a small sample of tissue is removed from the jugular vein and examined under a microscope to confirm the presence of a glomus jugulare tumor.

If a glomus jugulare tumor is diagnosed, treatment may involve one or more of the following:

1. Surgery to remove the tumor
2. Embolization, in which a small catheter is inserted into the jugular vein and a substance is injected to block the blood flow to the tumor
3. Radiation therapy, in which high-energy rays are used to kill the cancer cells
4. Chemotherapy, in which drugs are used to kill the cancer cells.

It's important to note that each case is unique and the treatment plan will be tailored to the individual patient's needs. The best course of treatment will depend on the location, size, and aggressiveness of the tumor, as well as other factors such as the patient's overall health and medical history.

Types of Peripheral Nerve Injuries:

1. Traumatic Nerve Injury: This type of injury occurs due to direct trauma to the nerve, such as a blow or a crush injury.
2. Compression Neuropathy: This type of injury occurs when a nerve is compressed or pinched, leading to damage or disruption of the nerve signal.
3. Stretch Injury: This type of injury occurs when a nerve is stretched or overstretched, leading to damage or disruption of the nerve signal.
4. Entrapment Neuropathy: This type of injury occurs when a nerve is compressed or trapped between two structures, leading to damage or disruption of the nerve signal.

Symptoms of Peripheral Nerve Injuries:

1. Weakness or paralysis of specific muscle groups
2. Numbness or tingling in the affected area
3. Pain or burning sensation in the affected area
4. Difficulty with balance and coordination
5. Abnormal reflexes
6. Incontinence or other bladder or bowel problems

Causes of Peripheral Nerve Injuries:

1. Trauma, such as a car accident or fall
2. Sports injuries
3. Repetitive strain injuries, such as those caused by repetitive motions in the workplace or during sports activities
4. Compression or entrapment of nerves, such as carpal tunnel syndrome or tarsal tunnel syndrome
5. Infections, such as Lyme disease or diphtheria
6. Tumors or cysts that compress or damage nerves
7. Vitamin deficiencies, such as vitamin B12 deficiency
8. Autoimmune disorders, such as rheumatoid arthritis or lupus
9. Toxins, such as heavy metals or certain chemicals

Treatment of Peripheral Nerve Injuries:

1. Physical therapy to improve strength and range of motion
2. Medications to manage pain and inflammation
3. Surgery to release compressed nerves or repair damaged nerves
4. Electrical stimulation therapy to promote nerve regeneration
5. Platelet-rich plasma (PRP) therapy to stimulate healing
6. Stem cell therapy to promote nerve regeneration
7. Injection of botulinum toxin to relieve pain and reduce muscle spasticity
8. Orthotics or assistive devices to improve mobility and function

It is important to seek medical attention if you experience any symptoms of a peripheral nerve injury, as early diagnosis and treatment can help prevent long-term damage and improve outcomes.

Causes:

1. Refractive errors: Diplopia can be caused by refractive errors such as myopia (nearsightedness), hyperopia (farsightedness), astigmatism, or presbyopia (age-related loss of near vision).
2. Eye alignment problems: Disorders such as strabismus (crossed eyes) or esotropia (eyes turned inward) can cause diplopia.
3. Cataracts: A cataract can cause diplopia due to the clouding of the lens in one or both eyes.
4. Glaucoma: Diplopia can be a symptom of glaucoma, a group of eye conditions that damage the optic nerve.
5. Retinal detachment: A retinal detachment can cause diplopia due to the separation of the retina from the underlying tissue.
6. Brain injuries or disorders: Diplopia can be a result of brain injuries or disorders such as stroke, traumatic brain injury, or multiple sclerosis.
7. Medications: Certain medications such as antidepressants, anti-seizure drugs, and chemotherapy drugs can cause diplopia as a side effect.

Diagnosis:

To diagnose diplopia, an eye examination is necessary. The doctor may perform a cover test to determine the type of diplopia and rule out other conditions. Imaging tests such as ultrasound or MRI may also be ordered to examine the eye and surrounding tissues.

Treatment:

The treatment of diplopia depends on the underlying cause. In some cases, glasses or contact lenses can correct refractive errors and alleviate symptoms. Surgery may be necessary for eye alignment problems such as strabismus or cataracts. In cases where the condition is caused by a brain disorder or injury, treatment of the underlying condition can resolve diplopia.

Prognosis:

The prognosis for diplopia varies depending on the underlying cause. In some cases, the condition may resolve on its own or with simple correction such as glasses or contact lenses. In other cases, surgery or treatment of an underlying condition may be necessary to resolve diplopia. In rare cases, the condition can lead to complications such as amblyopia (lazy eye) or vision loss if left untreated.

Prevention:

Preventing diplopia is not always possible, but early detection and treatment of underlying conditions can help prevent complications and improve outcomes. Regular eye exams and monitoring of vision can also help detect diplopia early on. In some cases, prism lenses or glasses with a specific prescription may be recommended to alleviate symptoms and prevent progression of the condition.

In conclusion, diplopia is a common condition that can have various causes and underlying mechanisms. Early diagnosis and treatment are crucial to prevent complications and improve outcomes. Regular eye exams and monitoring of vision can help detect diplopia early on, and in some cases, simple correction such as glasses or contact lenses may be sufficient to resolve the condition. In other cases, surgery or treatment of an underlying condition may be necessary. With appropriate management, most people with diplopia can achieve good visual acuity and quality of life.

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Diseases of the Seventh Cranial Nerve". In Dyck PJ, Thomas PK (eds.). Peripheral Neuropathy (Fourth ed.). Philadelphia: W.B. ... Facial nerve". In Barral JP, Croibier A (eds.). Manual Therapy for the Cranial Nerves. Edinburgh: Churchill Livingstone. pp. ... The facial nerve passes close to the mastoid process. The inner surface of the mastoid portion presents a deep, curved groove, ...
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"Simultaneous involvement of third and sixth cranial nerve in a patient with Lyme disease". Neuroradiology. 45 (2): 85-7. doi: ... Magnetic resonance imaging allows direct visualization of the cranial nerves. Furthermore, it provides significant information ... Damage to the peripheral nerves, along with injury to the chorda tympani branch of the facial nerve, also cause dysgeusia. A ... For example, the blink reflex may be used to evaluate the integrity of the trigeminal nerve-pontine brainstem-facial nerve ...
"Cranial nerve involvement in CMT disease type 1 due to early growth response 2 gene mutation". Neurology. 54 (8): 1696-8. doi: ... partial fusion of the trigeminal nerve (V) with the facial (VII) and auditory (VII) nerves, the proximal nerve roots coming off ... Mutations in this gene are associated with the autosomal dominant Charcot-Marie-Tooth disease, type 1D, Dejerine-Sottas disease ... It is later expressed in the neural crest derived cells of the cranial ganglion. The protein encoded by Krox20 contains two ...
Glossopharyngeal nerve". Manual Therapy for the Cranial Nerves. Churchill Livingstone. pp. 181-189. doi:10.1016/B978-0-7020- ... doi:10.1016/B978-0-12-410390-0.00027-5. ISBN 978-0-12-410390-0. Thomas, P. K.; Mathias, Christopher J. (2005). "52 - Diseases ... The tympanic nerve is also known as the nerve of Jacobson, or Jacobson's nerve. Lesser petrosal nerve Sympathetic connections ... The tympanic nerve (Jacobson's nerve) is a branch of the glossopharyngeal nerve found near the ear. It gives sensation to the ...
Thomas, P. K.; Mathias, Christopher J. (2005). "52 - Diseases of the Ninth, Tenth, Eleventh, and Twelfth Cranial Nerves". ... Brandmeir, Nicholas (2015). "32 - Nerve Injuries of the Neck". Nerves and nerve injuries: Pain, treatment, injury, disease and ... This nerve branches from the first cervical nerve as it joins the hypoglossal nerve for a short distance. The thyrohyoid muscle ... It is supplied by the hypoglossal nerve, and a branch of the ventral rami of the cervical plexus, spinal nerve C1, which ...
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Sowka JW, Gurwood AS, Kabat AG (2000-2001). "Cranial Nerve VI Palsy". Handbook of Ocular Disease Management. Jobson Publishing ... Sixth nerve palsy, or abducens nerve palsy, is a disorder associated with dysfunction of cranial nerve VI (the abducens nerve ... fibers of the seventh cranial nerve wrap around the VIth nerve nucleus, and, if this is also affected, a VIth nerve palsy with ... The unilateral abducens nerve palsy is the most common of the isolated ocular motor nerve palsies. The nerve dysfunction ...
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... thrombosis and nerve compression syndrome of cranial nerves XI and XII. One death case appeared in the scientific literature, ... Additionally iron deposition occurs in different neurological diseases such as Alzheimer's disease or Parkinson's disease that ... Finally, an organ-specific immune response is not seen in any other kind of venous disease. Iron deposition as a cause of MS ... MS is also more common in women, while venous diseases are more common in men. Venous pathology is commonly associated with ...
The vagus nerve provides efferent nervous signals out from the hunger and satiety centers of the hypothalamus, a region of the ... Vagotomy is an essential component of surgical management of peptic (duodenal and gastric) ulcer disease (PUD). Vagotomy was ... is sometimes injured by ongoing treatment for acute lymphoblastic leukemia or surgery or radiation to treat posterior cranial ... "Could nerve-snipping spur weight loss? - USATODAY.com". USA Today. 2007-07-02. Retrieved 2010-05-27. Lustig, Robert H.; Pamela ...
... is a general term describing disease affecting the peripheral nerves, meaning nerves beyond the brain and spinal cord. Damage ... Brachial neuritis Cranial neuritis such as Bell's palsy Optic neuritis Vestibular neuritis Wartenberg's migratory sensory ... Genetic diseases: Friedreich's ataxia, Fabry disease, Charcot-Marie-Tooth disease, hereditary neuropathy with liability to ... Vitamin B6 excess Those with diseases or dysfunctions of their nerves may present with problems in any of the normal nerve ...
... damage to the inferior alveolar nerve occurs in 3.5% of mandibular distraction, tooth bud injury in 2%, and facial nerve injury ... Drugs and Diseases. Rossini G, Vinci B, Rizzo R, Pinho TM, Deregibus A (November 2016). "Mandibular distraction osteogenesis: a ... Introduction of Multidirectional Cranial Distraction Osteogenesis". Journal of Korean Neurosurgical Society. 59 (3): 233-41. ... Medscape Emedicine Drugs and Diseases. Retrieved 31 October 2016. Patel PK, Zhao L, Ellis MF (January 6, 2015). de la Torre JI ...
"The foramen is probed with a nerve hook to ensure that the nerve is free". To decompress a longer part of the cervical canal a ... The extent of the slot should not exceed half of the vertebral body - cranial or caudal, but at the same time is providing more ... "Intervertebral Disk Disease". cal.vet.upenn.edu. Retrieved 2019-12-02. Cloward, Ralph B. (November 1958). "The Anterior ... Attention is paid on any deep nerve structures as the recurrent laryngeal nerve. The goal is to expose the affected disc and ...
Dizziness Loss of sensation Cranial nerve disturbance Loss of the ability to know how joints are positioned Lhermitte's sign (' ... It also occurs in patients with bone diseases, such as osteomalacia, rheumatoid arthritis, Paget's disease, Ehlers-Danlos ... If the condition develops after birth, it is usually the result of injury or diseases. If due to injury, about half the time it ... Symptoms vary depending on whether the spinal cord, brain stem, nerves, or blood supply is affected by the pressure.[citation ...
All of which are present in the cortex, medulla, hippocampus, and motor cells of the spinal cord and XIIth cranial nerve. In ... 2008). "VCP disease associated with myopathy, Paget disease of bone and frontotemporal dementia: review of a unique disorder" ( ... There have been numerous advances in descriptions of genetic causes of FTLD, and the related disease amyotrophic lateral ... Examples of FTLD-tau include; Pick's disease, corticobasal degeneration, progressive supranuclear palsy. FTLD-TDP (or FTLD-U ) ...
Cranial nerve palsies occur in some unusual cases. In the bestselling 1996 non-fiction book Into Thin Air: A Personal Account ... Progress in Cardiovascular Diseases. 52 (6): 467-484. doi:10.1016/j.pcad.2010.02.003. PMID 20417340. Rosenberg, Gary (2012). ... but the increase in brain volume from edema does not likely cause cranial vault impingement. The presence of large sulci ...
Cranial nerve palsies and depressed tendon reflexes have also been reported.[citation needed] Studies have suggested that ... countries and eventually world-level organizations banned the insecticide for all purposes except for combating disease-vector ... "Atropine availability as an antidote for nerve agent casualties: Validated rapid reformulation of high-concentration atropine ...
... characterized by an overgrowth of the facial and cranial bones. It is not a disease in itself, but a symptom of other diseases ... Exophthalmos gradually develops, going on later to a complete loss of sight due to compression of the optic nerve by the ... In the somewhat less common form of this rare disease the overgrowth of bone affects all the cranial bones as well as those of ... Lee VS, Webb MS Jr, Martinez S, McKay CP, Leight GS Jr (April 1996). "Uremic leontiasis ossea: "bighead" disease in humans? ...
Its nerve fibres, the olivocochlear bundle (OCB), form part of the vestibulocochlear nerve (VIIIth cranial nerve, also known as ... presented data from eight patients who had undergone unilateral vestibular neurectomy to treat Ménière's disease, a procedure ... Winslow and Sachs (1987) found that stimulating the OCB: "...enables auditory nerve fibres to signal changes in tone level with ... These studies have measured the output of the auditory nerve (AN), with and without OCB stimulation. In 1956, Galambos ...
Diagnosis can be made via combination of physical exam, particularly deficits of the relevant cranial nerves. Confirmation can ... Vertical supranuclear ophthalmoplegia has also been associated with metabolic disorders, such as Niemann-Pick disease, Wilson's ... origin of cranial nerve III) and Edinger-Westphal nuclei, causing dysfunction to the motor function of the eye. Classically, it ... oculomotor nerve palsy, trochlear nerve palsy and internuclear ophthalmoplegia. Parinaud's syndrome results from injury, either ...
2019), who interpret their findings as indicative of the occurrence of a metabolic bone disease in the Cretan deer population, ... D. Rex Mitchell (2019). "The anatomy of a crushing bite: The specialised cranial mechanics of a giant extinct kangaroo". PLOS ... sinuses and endocranial nerves and vessels of the periptychid Carsioptychus coarctatus is published by Cameron et al. (2019). ... 2019). A study on the musculoskeletal diseases of Pleistocene sloths from the Brazilian Intertropical Region is published by ...
Centers for Disease Control and Prevention (CDC) (28 October 1994). "Human Rabies - Miami, 1994". Morbidity and Mortality ... Overstimulation of the photoreceptors in the retina Excessive electric impulses to the optic nerve Excessive response in the ... where photophobia can sometimes precede the clinical diagnosis by years Subarachnoid haemorrhage Tumor of the posterior cranial ... Centers for Disease Control and Prevention. 43 (42): 773-5. PMID 7935313. SCDS Society Bailey, Gretchyn. "Photophobia (Light ...
The relevant cranial nerves (specifically the oculomotor, trochlear, and abducens), as in cavernous sinus syndrome or raised ... The orbit of the eye, including mechanical restrictions of eye movement, as in Graves' disease. The muscle, as in progressive ... The brainstem nuclei of these nerves, as in certain patterns of brainstem stroke such as Foville's syndrome. White matter ... It is a physical finding in certain neurologic, ophthalmologic, and endocrine disease. Internal ophthalmoplegia means ...
Bilateral 6th cranial nerve palsies may occur, causing abnormalities related to eye movement, but this is rare. 40% of people ... a kidney problem causing protein loss in the urine Chronic inflammatory diseases, such as inflammatory bowel disease, lupus and ... The disease may be complicated by raised intracranial pressure, which may warrant surgical intervention such as the placement ... On 13 April 2021 the Centers for Disease Control and Prevention paused the use of the Janssen COVID-19 vaccine in the United ...
Evaluation of the cranial MRI screening of the patient with the misdoubt of cerebrovascular disease showed results that were ... Horner's syndrome is the disruption of a nerve pathway from the brain to the face and eye on one side of the body. This also ... During cranial examination there was flattening of left sided nasolabial sulcus with abnormal gag reflex observed. In the motor ... Cranial MRI showed a right hemi medullary infarct, and magnetic resonance angiography showered severe stenosis of the right ...
In blunt trauma, the facial nerve is the most commonly injured cranial nerve. Physical trauma, especially fractures of the ... "Lyme disease rashes and look-alikes". Lyme Disease. Centers for Disease Control and Prevention. Retrieved 18 April 2019. Wright ... the facial nerve can be reinnervated with techniques such as cross-facial nerve grafting, nerve transfers and end-to-end nerve ... Moebius syndrome is a bilateral facial paralysis resulting from the underdevelopment of the VII cranial nerve (facial nerve), ...
While vagal nerve stimulation is often a target area for treatment of epileptic seizures, there has been research into the ... This results in higher penetration depths (and therefore easier communication with the sub-cranial communicator), as well as ... Closed-loop cortical neuromodulation has also been investigated as a treatment technique for Parkinson's disease Warneke, B.; ... Neural dust is a term used to refer to nanometer-sized devices operated as wirelessly powered nerve sensors; it is a type of ...
Optic atrophy and cranial nerve damage secondary to bony expansion can result in marked morbidity. The prognosis is extremely ... It has been shown to provide long-term disease-free periods for a significant percentage of those treated. It can impact both ... Cranial nerve dysfunction and neurologic complications are usually associated with infantile osteopetrosis. Expansion of the ... Camurati-Engelmann disease) SOST-related sclerosing bone dysplasias The only effective line of treatment for malignant ...
A natural cast of the posterior brain, skull vessels and nerves, and the inner ear of Dinilysia patagonica is described by ... Paul V. Ullmann; Zachary M. Boles; Michael J. Knell (2018). "Insights into cranial morphology and intraspecific variation from ... evidence of injury and disease in fossil 'fish lizards'". Journal of Zoology. 304 (1): 21-33. doi:10.1111/jzo.12517. Alexandra ... A study on the anatomy of the brain, inner ear, nasal cavity and skull nerves of Proganochelys quenstedti, and on its ...
Alzheimer's disease, for example, is much more likely to develop in a person who has experienced a head injury. Brain damage, ... CT scans can show brain bleeds, fractures of the skull, fluid build up in the brain that will lead to increased cranial ... maxillar fracture an eye that cannot move or is deviated to one side can indicate that a broken facial bone is pinching a nerve ... Disease Control Priorities. Vol. 1 (3rd ed.). Washington, DC: World Bank. doi:10.1596/978-1-4648-0346-8. hdl:10986/21568. ISBN ...
Diseases : Arnold-Chiari Malformation, Chiaris Syndrome, Cranial Nerve Diseases. Therapeutic Actions : Applied Kinesiology, ... 1 Abstracts with Cranial Nerve Diseases Research. Filter by Study Type. Human Study. ... 2 Therapeutic Actions Researched for Cranial Nerve Diseases Name. AC. CK. Focus. ... kinesiology chiropractic treatment may have therapeutic value in symptomatic Arnold-Chiari malformation and cranial nerve ...
Asymmetric Type F Botulism with Cranial Nerve Demyelination. Emerging Infectious Diseases. 2012;18(1):102-104. doi:10.3201/ ... Asymmetric Type F Botulism with Cranial Nerve Demyelination [PDF - 311 KB] A. Filozov et al. View Abstract. Cite This Article. ... Cranial nerve demyelination was found during autopsy. Bilateral, asymmetric clinical signs, although rare, do not rule out ... Demyelination of cranial nerves might be underrecognized during autopsy of botulism patients. ...
Paralysis of the Third Cranial Nerve. Paralysis of the Fourth Cranial Nerve. Paralysis of the Fifth Cranial Nerve. Paralysis of ... Paralysis of the Seventh Cranial Nerve. Paralysis of the Eighth Cranial Nerve. Paralysis of the Ninth Cranial Nerve. Local ... Advances in neuroscience and neuroendocrinology have identified the brains hormones, nerve chemicals, and nerve pathways ... "Cure of Disease Without Drugs or Medicines," Harpers Weekly, 1889. Courtesy National Library of Medicine. Dr. Huber Dry Cell ...
Provided by the U.S. Centers for Disease Control and Prevention. ... Cranial nerve involvement: When the cranial nerves are affected ... What is neurologic Lyme disease?. Neurologic symptoms of Lyme disease occur when the Lyme disease bacteria affect the ... Peripheral nerve involvement: When the peripheral nerves are affected, patients can develop radiculoneuropathy which can cause ... Content source: Centers for Disease Control and Prevention, National Center for Emerging and Zoonotic Infectious Diseases ( ...
7. Monoparesis (monoplegia) and cranial mononeuropathy. Peripheral/cranial nerve and nerve root diseases.. Kornegay JN. Probl ... Primary sciatic nerve lymphoma: a case report and review of the literature.. Descamps MJ; Barrett L; Groves M; Yung L; Birch R ... 1. B-cell lymphoma in the peripheral nerves of a cat.. Higgins MA; Rossmeisl JH; Saunders GK; Hayes S; Kiupel M. Vet Pathol; ... A Case of Feline T-cell Lymphoma with Tropism for Striated Muscle and Peripheral Nerve.. Mori M; Izawa T; Sasaki H; Sonoyama J ...
Brain Diseases. Central Nervous System Diseases. Movement Disorders. Ophthalmoplegia. Ocular Motility Disorders. Cranial Nerve ... To assess disease severity in PSP [ Time Frame: 5 days ]. To assess disease severity in PSP subjects by SUVR as Assessed by 18F ... To assess disease progression in PSP [ Time Frame: 1.5 year ]. To assess disease progression in PSP subjects by SUVR as ... Major systemic diseases including coronary arterial disease, heart failure, uremia, hepatic failure, prominent strokes, acute ...
Diseases of the first cranial (olfactory) nerve, which usually feature anosmia or other alterations in the sense of smell and ... "Olfactory Nerve Diseases" by people in this website by year, and whether "Olfactory Nerve Diseases" was a major or minor topic ... "Olfactory Nerve Diseases" is a descriptor in the National Library of Medicines controlled vocabulary thesaurus, MeSH (Medical ... Below are the most recent publications written about "Olfactory Nerve Diseases" by people in Profiles. ...
The disease is transmitted to humans via tick bites, from infected ticks of the genus Ixodes. ... Lyme disease is a multisystem illness caused by infection with the spirochete Borrelia burgdorferi and the bodys immune ... Cranioneuropathy, especially cranial nerve VII and Bell palsy (peripheral seventh nerve palsy with decreased unilateral ... Signs and symptoms of Lyme disease vary by disease stage. Physical findings in patients with early disease are as follows:. * ...
Abnormal ocular motility is a common clinical feature in congenital cranial dysinnervation disorder (CCDD). To date, eight ... Oculomotor Nerve Diseases / genetics* * Phenotype * Tubulin / genetics * Tubulin / metabolism Substances * COL25A1 protein, ... Recessive mutations in COL25A1 are a cause of congenital cranial dysinnervation disorder Am J Hum Genet. 2015 Jan 8;96(1):147- ... Abnormal ocular motility is a common clinical feature in congenital cranial dysinnervation disorder (CCDD). To date, eight ...
Nervous System Diseases [C10]. *Cranial Nerve Diseases [C10.292]. *Optic Nerve Diseases [C10.292.700] ... Orbital Inflammation in Myelin Oligodendrocyte Glycoprotein Antibody-Associated Disease: A Case Report and Review of the ...
Cranial Nerves (1966-1980). Peripheral Nerve Diseases (1966-1980). specific cranial nerve (1966-1980). See Also. Meningitis, ... CRANIAL NERVE DIS. Entry Term(s). Cranial Nerve Disorders Cranial Nerve Palsies Cranial Neuropathies Cranial Neuropathies, ... Cranial Nerve Diseases Preferred Concept UI. M0005279. Scope Note. Disorders of one or more of the twelve cranial nerves. With ... Cranial Nerve Palsies Narrower Concept UI. M0360800. Terms. Cranial Nerve Palsies Preferred Term Term UI T369873. Date11/03/ ...
Cranial nerves. *. Dementia (35). *. Drugs: CNS (not psychiatric). *. Epilepsy and seizures (236) ...
Cranial nerve paralysis Ectopic ossification in muscle tissue Failure to thrive Hyperostosis Increased bone mineral density ... Many rare diseases have limited information. Currently GARD aims to provide the following information for this disease:. * ... The National Library of Medicines Unified Medical Language System (UMLS) is used to classify and organize diseases and disease ... Symptoms of this disease may start to appear at any time in life.. The age symptoms may begin to appear differs between ...
The toxin causes a demyelinating polyneuropathy that affects cranial and peripheral nerves. The toxic effects usually begin ... Centers for Disease Control and Prevention (CDC): Infectious Diseases Related to Travel: Diphtheria ... The use of antitoxin for cutaneous disease, without evidence of respiratory disease, is of questionable value because toxic ... Mild disease with a serosanguineous or purulent discharge and irritation of the external nares and upper lip occur in patients ...
This disease results in facial droop and a palsy of this cranial nerve ... Parkinsons disease results from a deficiency of this neurotransmitter and is treated with this drug ... This the MOA for cholinesterase inhibitors in the treatment of Alzheimers disease ...
Behcets disease can affect many parts of the body, including the brain and spinal cord. ... Behcets disease is a rare disease that causes chronic inflammation, or swelling, in the bodys blood vessels. ... Cranial nerve palsies - the lack of function of nerves that lead from the brain to parts of the head, face, and trunk ... What is Behcets disease? Behcets disease is a rare disease that causes chronic inflammation, or swelling, in the bodys blood ...
... is a rare multisystem autoimmune disease of unknown etiology. Its hallmark features include necrotizing granulomatous ... manifesting with extensive cranial nerve palsies and cranial diabetes insipidus: a case report and literature review. BMC ... Kidney disease is present in 17% of patients at initial diagnosis and is usually asymptomatic. [4] Kidney failure occurs in 11 ... Drugs & Diseases , Rheumatology Granulomatosis with Polyangiitis (GPA, formerly Wegener Granulomatosis) Clinical Presentation. ...
Cranial Nerve Disease. Cranial Nerve Disorder. Cranial Nerve Disorders. Cranial Nerve Palsies. Cranial Nerve Palsy. Cranial ... Cranial Nerve Diseases Entry term(s). Cranial Nerve Disease Cranial Nerve Disorder Cranial Nerve Disorders Cranial Neuropathies ... Neuropathies, Multiple Cranial. Neuropathy, Cranial. Neuropathy, Multiple Cranial. Palsies, Cranial Nerve. Palsy, Cranial Nerve ... Cranial Nerves (1966-1980). Peripheral Nerve Diseases (1966-1980). specific cranial nerve (1966-1980). ...
Cranial Nerves (1966-1980). Peripheral Nerve Diseases (1966-1980). specific cranial nerve (1966-1980). See Also. Meningitis, ... CRANIAL NERVE DIS. Entry Term(s). Cranial Nerve Disorders Cranial Nerve Palsies Cranial Neuropathies Cranial Neuropathies, ... Cranial Nerve Diseases Preferred Concept UI. M0005279. Scope Note. Disorders of one or more of the twelve cranial nerves. With ... Cranial Nerve Palsies Narrower Concept UI. M0360800. Terms. Cranial Nerve Palsies Preferred Term Term UI T369873. Date11/03/ ...
... cranial nerve [CN] X) is the longest cranial nerve in the body, containing both motor and sensory functions in both the ... Because of the wide distribution of the nerve throughout the body, there are several clinical correlations of the vagus nerve. ... The nerve travels widely throughout the body affecting several organ systems and regions of the body, such as the tongue, ... in patients with chronic obstructive airway disease and congestive heart failure. One of its dysfunctions could also be a ...
It is a chronic disease and may be monophasic, progressive or recurrent with exacerbations and incomplete remissions, causing ... Activated Th1 and Th2 cells induce inflammation at the spinal roots, causing nerve roots hypertrophy, affection of the cranial ... It is a chronic disease and may be monophasic, progressive or recurrent with exacerbations and incomplete remissions, causing ... IgG4 anti-NF155 antibodies move into places where the blood-nerve barrier is absent or leaky, such a nerve terminal and nerve ...
... cranial nerve palsy, hydrocephalus, cardiac defects and congenital heart disease, polydactyly, deformities of toes, ... cerebrovascular disease, serious heart disease, anemia, malignancy, trauma, renal insufficiency, concomitant drugs (see ... cerebrovascular disease, serious heart disease, anemia, malignancy, trauma, renal insufficiency, concomitant drugs (see ... Infectious diseases or disturbances of intestinal fora: sprue, antibiotic therapy.. Trauma which may result in internal ...
Disease Prevention and Treatment Subcommittee Report to the Tick-Borne Working Group ... cranial nerve palsy, and myocarditis. It is unclear whether these other complications occur in B. miyamotoi infections. ... Tick-Borne Disease Working Group * About the Tick-Borne Disease Working Group * 21st Century Cures Act ... Treatment of Lyme Disease. Yes. 6. Charlotte Mao, MD, MPH, Dean Center for Tick Borne Illness. Treatment: Lyme Disease and ...
The zygomaticus major muscle receives nerves from cranial nerve VII. Conditions that may affect the zygomaticus major muscle ... include myalgia strains, neuromuscular diseases, lacerations, contusions, Bells palsy, infectious myositis, and myopathy. ...
Provided by the Centers for Disease Control and Prevention (CDC). ... and cranial nerve palsies. People at risk for more severe ... Chikungunya virus disease is a notifiable disease in the United States, and cases should be reported to state, territorial, and ... The Centers for Disease Control and Prevention (CDC) protects peoples health and safety by preventing and controlling diseases ... The Centers for Disease Control and Prevention (CDC) is issuing this Health Alert Network (HAN) Health Advisory to notify ...
Cavernous sinus and jugular thromboses, base of skull osteomyelitis and cranial nerve palsies: catastrophic complications of ... Cavernous sinus and jugular thromboses, base of skull osteomyelitis and cranial nerve palsies: catastrophic complications of ...
... stage of disease, focal weakness, cranial nerve palsy, and hydrocephalus. [15] Children with advanced disease with neurological ... As TBM has a predilection for the skull base (unlike bacterial meningitis), cranial nerve palsies are more common. CN VI is ... Moghtaderi A, Alavi-Naini R, Rashki S. Cranial nerve palsy as a factor to differentiate tuberculous meningitis from acute ... Official American Thoracic Society/Centers for Disease Control and Prevention/Infectious Diseases Society of America Clinical ...
Dive into the research topics of Pupil-Involving Third Cranial Nerve Paresis at High Altitude. Together they form a unique ... Abducens Nerve Diseases 34% * Cranial Nerve Diseases 31% * Neuroimaging 21% * Oceans and Seas 27% ...
  • PNS manifestations include mononeuritis multiplex, sensorimotor polyneuropathy, and cranial nerve palsies. (medscape.com)
  • When the cranial nerves are affected, facial palsy (droop) can occur on one or both sides of the face. (cdc.gov)
  • Neurological complications most often occur in early disseminated Lyme disease, with numbness, pain, weakness, facial palsy/droop (paralysis of the facial muscles), visual disturbances, and meningitis symptoms such as fever, stiff neck, and severe headache. (cdc.gov)
  • Compression of the cranial nerves can lead to paralyzed facial muscles (facial nerve palsy), hearing loss, vision loss, and a sense of smell that is diminished (hyposmia) or completely absent (anosmia). (medlineplus.gov)
  • Bell's palsy is a disease process affecting the seventh cranial nerve leading to abrupt facial paralysis/weakness as well as the symptoms presented in the case study, which is usually unilateral and self-remitting within a few months to a year. (brainscape.com)
  • Other areas of the body are not affected with Bell's palsy, as would be evident with Lyme disease, Guillian-Barré syndrome, and stroke. (brainscape.com)
  • Serratus anterior palsy is a dysfunction that is characteristic of traumatic , non-traumatic, and idiopathic injury to the long thoracic nerve. (mdwiki.org)
  • Clivus and cervical spinal osteomyelitis with epidural abscess presenting with multiple cranial neuropathies, Clin. (manipal.edu)
  • Centers for Disease Control and Prevention. (cdc.gov)
  • The Centers for Disease Control and Prevention (CDC) cannot attest to the accuracy of a non-federal website. (cdc.gov)
  • courtesy of the US Centers for Disease Control and Prevention). (medscape.com)
  • Outbreaks in Indonesia, Thailand, Vietnam, Laos, South Africa, Sudan, and Pakistan have occurred since 2011 (travel information about diphtheria is available at the Centers for Disease Control and Prevention [CDC] web site for travelers' health ). (merckmanuals.com)
  • The Centers for Disease Control and Prevention (CDC) is issuing this Health Alert Network (HAN) Health Advisory to notify clinicians and public health authorities of an increase in the number of cases of chikungunya reported in Paraguay. (cdc.gov)
  • Lyme disease can be contracted anywhere in the UK … Centers for Disease Control and Prevention. (choconetchocolates.com)
  • They also made the first attempts to understand emotions as mental phenomena that had surprising and complex connections to the inner workings of bodily systems and their disorders in disease. (nih.gov)
  • Disorders of one or more of the twelve cranial nerves. (nih.gov)
  • With the exception of the optic and olfactory nerves, this includes disorders of the brain stem nuclei from which the cranial nerves originate or terminate. (nih.gov)
  • Patients with MM often have neurological complications, either due to metabolic disorders such as hypercalcaemia, uraemia and hyperviscosity or due to peripheral neuro-pathy, spinal cord compression and cranial nerve infiltration [5]. (who.int)
  • Unlike most forms of bacterial meningitis, TBM is more likely to cause neurological deficits, including altered mental status, personality changes, and, as the lesions may result in neurovascular compression, cranial nerve deficits and infarcts. (medscape.com)
  • Some research suggests a relationship to cranial trauma, benign cranial lesions, the herpes virus , a disease of the blood vessels in the brain, or nerve sensitization. (verywellhealth.com)
  • [1] This is typically due to damage (i.e. lesions ) of the long thoracic nerve . (mdwiki.org)
  • Olfactory Nerve Diseases" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, MeSH (Medical Subject Headings) . (umassmed.edu)
  • Diseases of the first cranial (olfactory) nerve, which usually feature anosmia or other alterations in the sense of smell and taste. (umassmed.edu)
  • This graph shows the total number of publications written about "Olfactory Nerve Diseases" by people in this website by year, and whether "Olfactory Nerve Diseases" was a major or minor topic of these publications. (umassmed.edu)
  • Below are the most recent publications written about "Olfactory Nerve Diseases" by people in Profiles. (umassmed.edu)
  • The vagus nerve (cranial nerve [CN] X) is the longest cranial nerve in the body, containing both motor and sensory functions in both the afferent and efferent regards. (nih.gov)
  • Because of the wide distribution of the nerve throughout the body, there are several clinical correlations of the vagus nerve. (nih.gov)
  • The vagus nerve has its origin in the medulla oblongata and exits the skull via the jugular foramen. (nih.gov)
  • the spinal accessory nerve (CN XI) joins the vagus nerve just distal to the inferior ganglion. (nih.gov)
  • The vagus nerve continues by traveling inferiorly within the carotid sheath where it is located posterior and lateral to the internal and common carotid arteries, and medial to the internal jugular vein. (nih.gov)
  • it makes its descent into the thoracic cavity by traveling to the right of the trachea, and posterior to the hilum on the right, moving medially to form the esophageal plexus with the left vagus nerve. (nih.gov)
  • it then descends posteriorly to the phrenic nerve and posterior to the left lung, then travels medially towards to the esophagus forming the esophageal plexus with the right vagus nerve. (nih.gov)
  • There are four branches of the vagus nerve within the neck: pharyngeal branches, superior laryngeal nerve, recurrent laryngeal nerve, and the superior cardiac nerve. (nih.gov)
  • The right recurrent laryngeal nerve's fibers branch from the vagus nerve near the right subclavian artery, traveling superiorly to enter the larynx between the cricopharyngeus muscle and the esophagus. (nih.gov)
  • The vagus nerve is the correct answer because it is the motor supply for the muscles of the pharynx (swallowing) and the larynx (voice). (brainscape.com)
  • Some may become confused because of the similarity of names of "hypoglossal" (cranial nerve XII, control of the tongue) and "glossopharyngeal" (cranial nerve IX, whose functions are paired with those of the vagus nerve). (brainscape.com)
  • The following terms were included: 'vagus nerve', '10. (emf-portal.org)
  • Characterization of vagus nerve stimulation-induced pupillary responses in epileptic patients [med. (emf-portal.org)
  • When the peripheral nerves are affected, patients can develop radiculoneuropathy which can cause numbness, tingling, "shooting" pain, or weakness in the arms or legs. (cdc.gov)
  • 1. B-cell lymphoma in the peripheral nerves of a cat. (nih.gov)
  • It is a significant characteristic in the diagnosis of all diseases, but it becomes more significant in conditions that do not have a clear mechanism of injury or causative etiology. (brainscape.com)
  • Antibiotic selection, route of administration, and duration of therapy for Lyme disease are guided by the patient's clinical manifestations and stage of disease, as well as the presence of any concomitant medical conditions or allergies. (medscape.com)
  • Abnormal ocular motility is a common clinical feature in congenital cranial dysinnervation disorder (CCDD). (nih.gov)
  • Consider participating in a clinical trial so clinicians and scientists can learn more about Behcet's disease. (nih.gov)
  • Clinical research uses human volunteers to help researchers learn more about a disorder and perhaps find better ways to safely detect, treat, or prevent disease. (nih.gov)
  • Learn about clinical trials currently looking for people with Behcet's disease at Clinicaltrials.gov , a database of current and past clinical studies and research results. (nih.gov)
  • For patients with suspected chikungunya disease, it is important to rule out dengue virus infection because proper clinical management of dengue can improve the outcome. (cdc.gov)
  • Currently, more than 2 billion people (ie, one third of the world's population) are infected with tuberculosis (TB), 10% of whom develop clinical disease, and 1.4 million of whom die of the disease annually. (medscape.com)
  • Many disease processes have premonitory symptoms that help identify the disease by the clinical pattern. (brainscape.com)
  • Clinical treatments may also cause injury to the long thoracic nerve ( iatrogenesis from forceful manipulation, mastectomies with axillary node dissection, surgical treatment of spontaneous pneumothorax , post-general anesthesia for various clinical reasons, and electrical shock, amongst others). (mdwiki.org)
  • Uveitis can be associated with many systemic diseases, and may in some cases be the first clinical sign observed. (vin.com)
  • The pharyngeal nerve branches arise from the inferior ganglion of CN X containing both sensory and motor fibers. (nih.gov)
  • A syndrome characterized by recurrent episodes of excruciating pain lasting several seconds or longer in the sensory distribution of the TRIGEMINAL NERVE. (wakehealth.edu)
  • Neurologic symptoms of Lyme disease occur when the Lyme disease bacteria affect the peripheral or central nervous systems. (cdc.gov)
  • Most people with Lyme disease respond well to antibiotics and fully recover. (cdc.gov)
  • Varying degrees of permanent nervous system damage may develop in people who do not receive treatment in the early stages of illness and who develop late-stage Lyme disease. (cdc.gov)
  • Neurologic symptoms do not necessarily indicate central nervous system infection in a patient with Lyme disease. (cdc.gov)
  • Two-step serologic testing for Lyme disease is the recommended diagnostic test for neurologic Lyme disease. (cdc.gov)
  • Halperin J. Nervous system Lyme disease: diagnosis and treatment external icon . (cdc.gov)
  • Lyme disease, the most common vector-borne illness in the United States, is a multisystem illness usually caused by infection with the spirochete Borrelia burgdorferi (see the image below) and the body's immune response to the infection. (medscape.com)
  • See Lyme Disease and 4 Emerging Tick-Borne Illnesses , a Critical Images slideshow, to help identify and treat several tick-borne conditions. (medscape.com)
  • Signs and symptoms of Lyme disease vary by disease stage. (medscape.com)
  • In July 2019, the US Food & Drug Administration (FDA) approved the use of concurrent or sequential EIA testing for diagnosis of Lyme disease. (medscape.com)
  • With appropriate antibiotic treatment, most patients with early-stage Lyme disease recover rapidly and completely. (medscape.com)
  • Care was made to include members with different perspectives on Lyme disease treatment, because that issue has consistently shown to generate divergent opinions in previous Tick-Borne Disease Working Group reports. (hhs.gov)
  • The 2018 Report to Congress focused primarily on Lyme disease, while the 2020 Report to Congress was expanded to include more discussion of the other TBDs. (hhs.gov)
  • Borrelia burgdorferi and Borrelia mayonii cause Lyme disease in the United States, while Borrelia afzelii and Borrelia garinii are the leading causes in Europe and Asia. (choconetchocolates.com)
  • Lyme disease is caused by Borrelia, a spirochete bacteria. (choconetchocolates.com)
  • Lyme disease can also affect humans if bitten by an infected tick. (choconetchocolates.com)
  • 3. Lyme disease is caused by four main species of bacteria. (choconetchocolates.com)
  • When a tick feeds on infected wildlife the tick picks up the bacteria, then Lyme disease is spread to dogs and cats through the bite of an infected tick. (choconetchocolates.com)
  • Symptoms of Lyme disease. (choconetchocolates.com)
  • Lyme disease is a bacterial infection transmitted by the deer tick. (choconetchocolates.com)
  • Lyme disease is a bacterial infection that can be spread to humans by infected ticks. (choconetchocolates.com)
  • Each dot represents one case of Lyme disease and is placed randomly in the patient's county of residence. (choconetchocolates.com)
  • Lyme disease is a tick-borne bacterial infection that can be transmitted to humans when they are bitten by an infected Ixodes ricinus tick. (choconetchocolates.com)
  • This animated map shows the spread of Lyme disease across the U.S. from 2001 to 2013. (choconetchocolates.com)
  • Lyme disease with focal symptoms. (choconetchocolates.com)
  • Aug. 26, 2019 - Saliva from a tick's bite can transmit pathogens that cause serious illnesses, such as Lyme disease, and significant agricultural losses. (choconetchocolates.com)
  • Lyme disease is emerging in Canada due to expansion of the range of the tick vector Ixodes scapularis from the United States. (choconetchocolates.com)
  • Map: CDC, GIF: Shan Wang for NECIR) Deer ticks - often no bigger than the size of a poppy seed - become more active as the weather heats up, latching onto pets and people as … The presence of a dot in a state does not necessarily mean that Lyme disease was acquired in that state. (choconetchocolates.com)
  • The south of England and the Scottish Highlands have been earmarked as high risk areas for Lyme disease. (choconetchocolates.com)
  • Body aches Lyme disease affecting the cranial nerves or peripheral nervous system. (choconetchocolates.com)
  • Many people with early symptoms of Lyme disease develop a circular red skin rash around a tick bite. (choconetchocolates.com)
  • Estimated risk areas for Lyme disease has expanded in Windsor-Essex for 2019 compared to 2018. (choconetchocolates.com)
  • On her last day as an HHS Working Group (WG) member, after serving 2 terms, Lyme Disease Association President Pat Smith addressed at the Dec. 2 online WG meeting, her concerns about the procedures of the 2019-2020 Working Group and her concern that the … It can vary from no illness to severe disease, and signs may start 1 to 2 weeks after infection (the tick bite). (choconetchocolates.com)
  • As the disease progresses, the Rich foci enlarge and may eventually rupture into the subarachnoid space, resulting in meningitis (See Pathophysiology ). (medscape.com)
  • OBJECTIVE: To analyze the psychological status in patients with trigeminal neuralgia before surgery and in the early postoperative period after microvascular decompression of the trigeminal nerve. (bvsalud.org)
  • Readers should not consider the report or any part of it to be guidance or instruction regarding the diagnosis, care, or treatment of tick-borne diseases or to supersede in any way existing guidance. (hhs.gov)
  • The Prevention and Treatment Subcommittee of the 2021-2022 Tick-Borne Disease Working Group convened to address advances and gaps in the prevention and treatment of tick-borne diseases (TBDs). (hhs.gov)
  • Muscle aches This conference brings together some of the leading medical professionals in the field of tick-borne diseases. (choconetchocolates.com)
  • therefore, damage to or impingement of this nerve can result in weakening or paralysis of the muscle. (mdwiki.org)
  • Symptoms of this disease may start to appear at any time in life. (nih.gov)
  • The age symptoms may begin to appear differs between diseases. (nih.gov)
  • The symptoms from some diseases may begin at any age. (nih.gov)
  • The types of symptoms experienced, and their intensity, may vary among people with this disease. (nih.gov)
  • Which of the following disease processes often presents with premonitory symptoms? (brainscape.com)
  • NIEHS research uses state-of-the-art science and technology to investigate the interplay between environmental exposures, human biology, genetics, and common diseases to help prevent disease and improve human health. (nih.gov)
  • 229 disease terms (MeSH) has been reported with NOD2 gene. (cdc.gov)
  • A patient states that he recently had a benign mass removed from his upper neck and that a nerve was damaged during the surgery. (brainscape.com)
  • Severe atrophy of the trapezius is seen with accidental damage to the spinal accessory nerve during lymph node biopsy of the neck. (mdwiki.org)
  • Affected individuals tend to have less severe cranial nerve compression, resulting in milder neurological features. (medlineplus.gov)
  • [1] The second category is the lateral winging which is caused by injury of the spinal accessory nerve . (mdwiki.org)
  • The temporal pattern of a disease or condition is more significant for distinguishing which classification of headaches? (brainscape.com)
  • Central nervous system (CNS) manifestations include vasculitis of small to medium-sized vessels of the brain or spinal cord and granulomatous masses that involve the orbit, optic nerve, meninges or brain. (medscape.com)
  • SOST mutations that cause van Buchem disease result in a shortage of functional sclerostin. (medlineplus.gov)
  • All types of volunteers are needed-those who are healthy or may have an illness or disease-of all different ages, sexes, races, and ethnicities to ensure that study results apply to as many people as possible, and that treatments will be safe and effective for everyone who will use them. (nih.gov)
  • It can vary from no illness to severe disease, and signs may start 1 to 2 weeks after infection (the tick bite). (choconetchocolates.com)
  • Nontoxigenic strains of C. diphtheriae can also cause nasopharyngeal infection and sometimes systemic disease (eg, endocarditis, septic arthritis). (merckmanuals.com)
  • It's the most common tick-borne disease in the northern hemisphere and there are multiple strains of the bacteria. (choconetchocolates.com)
  • Kidney disease is present in 17% of patients at initial diagnosis and is usually asymptomatic. (medscape.com)
  • Prompt diagnosis of underlying viral infection in cats with ophthalmic disease is paramount for accurate diagnosis and prognosis and is required for appropriate therapeutic decision-making. (vin.com)
  • Sinusitis and disease in the nasal mucosa are the most common findings. (medscape.com)
  • DESCRIPTION (provided by applicant): Craniosynostosis (CS), the premature fusion of one or more cranial sutures, is a common, major structural birth defect occurring in about 1 in 2,500 live births. (nih.gov)
  • PUBLIC HEALTH RELEVANCE: Nonsyndromic craniosynostosis (NCS) is a common, major structural birth defect - due to the premature fusion of one or more cranial sutures - that requires extensive surgical correction and is associated with considerable ongoing medical problems and health care costs. (nih.gov)
  • In recent reports, between 38% and 70% of the cats with uveitis have concurrent systemic disease. (vin.com)
  • The granulomatous form of FIP, which is caused by a partial cell-mediated immune response, is most often associated with ocular signs, and uveitis may even be present without concurrent signs of systemic disease. (vin.com)
  • This disease is caused by a change in the genetic material (DNA). (nih.gov)
  • The exact cause of Behcet's disease is unknown, but it is thought to be due to a combination of genetic, immune, and environmental factors. (nih.gov)
  • There are numerous ways in which the long thoracic nerve can sustain trauma-induced injury. (mdwiki.org)
  • It also highlights those at increased risk for severe disease and prevention measures to mitigate additional spread of the virus and potential importation into unaffected areas, including the United States. (cdc.gov)
  • Discoveries in a field of research that explores the connections between the nervous, endocrine, and immune systems confirm that emotions influence the onset and course of disease, and also show how this interaction works. (nih.gov)
  • Several possible causes have been linked to the onset or recurrence of the disease process, one of which is a recent viral infection. (brainscape.com)
  • Uveitis with or without chorioretinitis is one of the most frequent and significant ophthalmic diseases in cats. (vin.com)
  • When the headache stabs are fixed on one spot, a headache specialist first must rule out a cranial nerve problem or brain structural problem (e.g., a tumor) with imaging tests . (verywellhealth.com)
  • 18. A Case of Feline T-cell Lymphoma with Tropism for Striated Muscle and Peripheral Nerve. (nih.gov)
  • Behcet's disease is a rare disease that causes chronic inflammation, or swelling, in the body's blood vessels. (nih.gov)
  • It is a chronic disease and may be monophasic, progressive or recurrent with exacerbations and incomplete remissions, causing accumulating disability. (nih.gov)
  • Fewer than 1,000 people in the U.S. have this disease. (nih.gov)
  • How can I or my loved one help improve care for people with Behcet's disease? (nih.gov)
  • People with van Buchem disease are typically of average height and do not have syndactyly or nail abnormalities. (medlineplus.gov)
  • In people with van Buchem disease, the skeletal features typically appear in childhood or adolescence. (medlineplus.gov)
  • Van Buchem disease has been reported in approximately 30 people. (medlineplus.gov)
  • Most people with van Buchem disease are of Dutch ancestry. (medlineplus.gov)
  • This shortage reduces the protein's ability to inhibit bone formation, causing the excessive bone growth seen in people with van Buchem disease. (medlineplus.gov)
  • Peripheral nervous system (PNS) involvement may occur in as many as 67% of patients, typically later in the disease course. (medscape.com)
  • During the public meeting on August 26, 2021, the Tick-Borne Disease Working Group selected Dennis M. Dixon, a federal representative, and Sunil K. Sood, a public representative, to serve as co-chairs of the Disease Prevention and Treatment Subcommittee. (hhs.gov)
  • the dorsal motor nucleus of X, superior ganglion of X, and the inferior ganglion of X. The nerve fibers from the nucleus ambiguous are efferent, special visceral (ESV) fibers which help to mediate swallowing and phonation. (nih.gov)
  • Two of the case-patients had Crohn disease and had undergone previous bowel surgery, which may have contributed to infection with C. botulinum . (cdc.gov)
  • Behcet's disease can affect many parts of the body, including the brain and spinal cord. (nih.gov)
  • Wilson's disease [4] is a rare familial disorder inherited as an autosomal recessive trait, being characterised by excessive accumulation of copper in the body particularly the liver, brain, cornea and kidney. (jpgmonline.com)
  • Once a nerve or brain problem is ruled out, these ice pick headaches are referred to as primary stabbing headaches. (verywellhealth.com)

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