AnatomyDiseasesChemicals and DrugsPhenomena and ProcessesDisciplines and OccupationsAnthropology, Education, Sociology and Social PhenomenaTechnology, Industry, AgricultureHealth Care
ColestipolNiacinResins, PlantMethylhydrazinesPolyaminesLovastatinDimethylhydrazinesHyperlipoproteinemia Type IIHypolipidemic AgentsBlinkingEyelidsFuneral RitesPharmaciesMortuary PracticeCommercePharmacyCholesterolCholesterol, DietaryCholesterol, LDLCholesterol, HDLHeartCholesterol EstersCholesterol Oxidase
Colestipol
Highly crosslinked and insoluble basic anion exchange resin used as anticholesteremic. It may also may reduce triglyceride levels.
Niacin
A water-soluble vitamin of the B complex occurring in various animal and plant tissues. It is required by the body for the formation of coenzymes NAD and NADP. It has PELLAGRA-curative, vasodilating, and antilipemic properties.
Resins, Plant
Flammable, amorphous, vegetable products of secretion or disintegration, usually formed in special cavities of plants. They are generally insoluble in water and soluble in alcohol, carbon tetrachloride, ether, or volatile oils. They are fusible and have a conchoidal fracture. They are the oxidation or polymerization products of the terpenes, and are mixtures of aromatic acids and esters. Most are soft and sticky, but harden after exposure to cold. (From Grant & Hackh's Chemical Dictionary, 5th ed & Dorland, 28th ed)
Methylhydrazines
Hydrazines substituted by one or more methyl groups in any position.
Polyamines
Lovastatin
A fungal metabolite isolated from cultures of Aspergillus terreus. The compound is a potent anticholesteremic agent. It inhibits 3-hydroxy-3-methylglutaryl coenzyme A reductase (HYDROXYMETHYLGLUTARYL COA REDUCTASES), which is the rate-limiting enzyme in cholesterol biosynthesis. It also stimulates the production of low-density lipoprotein receptors in the liver.
Dimethylhydrazines
Hydrazines substituted with two methyl groups in any position.
Hyperlipoproteinemia Type II
A group of familial disorders characterized by elevated circulating cholesterol contained in either LOW-DENSITY LIPOPROTEINS alone or also in VERY-LOW-DENSITY LIPOPROTEINS (pre-beta lipoproteins).
Hypolipidemic Agents
Substances that lower the levels of certain LIPIDS in the BLOOD. They are used to treat HYPERLIPIDEMIAS.
Blinking
Brief closing of the eyelids by involuntary normal periodic closing, as a protective measure, or by voluntary action.
Eyelids
Each of the upper and lower folds of SKIN which cover the EYE when closed.
Funeral Rites
Those customs and ceremonies pertaining to the dead.
Pharmacies
Facilities for the preparation and dispensing of drugs.
Mortuary Practice
Activities associated with the disposition of the dead. It excludes cultural practices such as funeral rites.
Commerce
The interchange of goods or commodities, especially on a large scale, between different countries or between populations within the same country. It includes trade (the buying, selling, or exchanging of commodities, whether wholesale or retail) and business (the purchase and sale of goods to make a profit). (From Random House Unabridged Dictionary, 2d ed, p411, p2005 & p283)
Pharmacy
The practice of compounding and dispensing medicinal preparations.
Cholesterol
The principal sterol of all higher animals, distributed in body tissues, especially the brain and spinal cord, and in animal fats and oils.
Cholesterol, Dietary
Cholesterol present in food, especially in animal products.
Cholesterol, LDL
Cholesterol which is contained in or bound to low density lipoproteins (LDL), including CHOLESTEROL ESTERS and free cholesterol.
Cholesterol, HDL
Cholesterol which is contained in or bound to high-density lipoproteins (HDL), including CHOLESTEROL ESTERS and free cholesterol.
Heart
The hollow, muscular organ that maintains the circulation of the blood.
Cholesterol Esters
Fatty acid esters of cholesterol which constitute about two-thirds of the cholesterol in the plasma. The accumulation of cholesterol esters in the arterial intima is a characteristic feature of atherosclerosis.
Cholesterol Oxidase
An enzyme that catalyzes the oxidation of cholesterol in the presence of molecular oxygen to 4-cholesten-3-one and hydrogen peroxide. The enzyme is not specific for cholesterol, but will also oxidize other 3-hydroxysteroids. EC 1.1.3.6.

Evidence for a new pathophysiological mechanism for coronary artery disease regression: hepatic lipase-mediated changes in LDL density. (1/41)

BACKGROUND: Small, dense LDL particles are associated with coronary artery disease (CAD) and predict angiographic changes in response to lipid-lowering therapy. Intensive lipid-lowering therapy in the Familial Atherosclerosis Treatment Study (FATS) resulted in significant improvement in CAD. This study examines the relationship among LDL density, hepatic lipase (HL), and CAD progression, identifying a new biological mechanism for the favorable effects of lipid-altering therapy. METHODS AND RESULTS: Eighty-eight of the subjects in FATS with documented coronary disease, apolipoprotein B levels >/=125 mg/dL, and family history of CAD were selected for this study. They were randomly assigned to receive lovastatin (40 mg/d) and colestipol (30 g/d), niacin (4 g/d) and colestipol, or conventional therapy with placebo alone or with colestipol in those with elevated LDL cholesterol levels. Plasma hepatic lipase (HL), lipoprotein lipase, and LDL density were measured when subjects were and were not receiving lipid-lowering therapy. LDL buoyancy increased with lovastatin-colestipol therapy (7.7%; P<0.01) and niacin-colestipol therapy (10.3%; P<0.01), whereas HL decreased in both groups (-14% [P<0.01] and -17% [P<0.01] with lovastatin-colestipol and niacin-colestipol, respectively). Changes in LDL buoyancy and HL activity were associated with changes in disease severity (P<0.001). In a multivariate analysis, an increase in LDL buoyancy was most strongly associated with CAD regression, accounting for 37% of the variance of change in coronary stenosis (P<0.01), followed by reduction in apolipoprotein Bl (5% of variance; P<0.05). CONCLUSIONS: These studies support the hypothesis that therapy-associated changes in HL alter LDL density, which favorably influences CAD progression. This is a new and potentially clinically relevant mechanism linking lipid-altering therapy to CAD improvement.  (+info)

Role of bile acids and bile acid binding agents in patients with collagenous colitis. (2/41)

BACKGROUND: In a retrospective study bile acid malabsorption was observed in patients with collagenous colitis. AIMS: To study the occurrence of bile acid malabsorption and the effect of bile acid binders prospectively in patients with chronic diarrhoea and collagenous colitis. METHODS: Over 36 months all patients referred because of chronic diarrhoea completed a diagnostic programme, including gastroscopy with duodenal biopsy, colonoscopy with biopsies, and the (75)Se-homocholic acid taurine ((75)SeHCAT) test for bile acid malabsorption. Treatment with a bile acid binder (cholestyramine in 24, colestipol in three) was given, irrespective of the results of the (75)SeHCAT test. RESULTS: Collagenous colitis was found in 28 patients (six men, 22 women), 27 of whom had persistent symptoms and completed the programme. Four patients had had a previous cholecystectomy or a distal gastric resection. The (75)SeHCAT test was abnormal in 12/27 (44%) of the collagenous colitis patients with (75)SeHCAT values 0.5-9.7%, and normal in 15 patients (56%). Bile acid binding treatment was followed by a rapid, marked, or complete improvement in 21/27 (78%) of the collagenous colitis patients. Rapid improvement occurred in 11/12 (92%) of the patients with bile acid malabsorption compared with 10/15 (67%) of the patients with normal (75)SeHCAT tests. CONCLUSION: Bile acid malabsorption is common in patients with collagenous colitis and is probably an important pathophysiological factor. Because of a high response rate without serious side effects, bile acid binding treatment should be considered for collagenous colitis, particularly patients with bile acid malabsorption.  (+info)

Common hepatic lipase gene promoter variant determines clinical response to intensive lipid-lowering treatment. (3/41)

BACKGROUND: The common -514 C-->T polymorphism in the promoter region of the hepatic lipase (HL) gene affects HL activity. The C allele is associated with higher HL activity, more dense and atherogenic LDL, and lower HDL(2) cholesterol. Intensive lipid-lowering therapy lowers HL activity, increases LDL and HDL buoyancy, and promotes coronary artery disease (CAD) regression. We tested the hypothesis that subjects with the CC genotype and a more atherogenic lipid profile experience the greatest CAD regression from these favorable effects. METHODS AND RESULTS: Forty-nine middle-aged men with dyslipidemia and established CAD who were undergoing intensive lipid-lowering therapy were studied. Change in coronary stenosis was assessed by quantitative angiography, HL polymorphism by polymerase chain reaction amplification, HL activity by (14)C-labeled substrate, and LDL buoyancy by density-gradient ultracentrifugation. The response to lipid-lowering therapy was significantly different among subjects with different HL promoter genotypes. Subjects with the C:C genotype had the greatest decrease in HL activity (P<0.005 versus TC and TT by ANOVA) and the greatest improvement in LDL density (P<0.005) and HDL(2)-C (P<0.05) with therapy. These subjects had the greatest angiographic improvement, with 96% of them experiencing CAD regression, compared with 60% of TC and none of the TT patients (P:<0.001). CONCLUSIONS: -In middle-aged men with established CAD and dyslipidemia, the HL gene -514 C-->T polymorphism significantly predicts changes in coronary stenosis with lipid-lowering treatment that appear to involve an HL-associated effect on LDL metabolism. This study identifies a gene polymorphism that strongly influences the lipid and clinical response to lipid-lowering drugs.  (+info)

Determinants of variable response to statin treatment in patients with refractory familial hypercholesterolemia. (4/41)

Interindividual variability in low density lipoprotein (LDL) cholesterol (LDL-C) response during treatment with statins is well documented but poorly understood. To investigate potential metabolic and genetic determinants of statin responsiveness, 19 patients with refractory heterozygous familial hypercholesterolemia were sequentially treated with placebo, atorvastatin (10 mg/d), bile acid sequestrant, and the 2 combined, each for 4 weeks. Levels of LDL-C, mevalonic acid (MVA), 7-alpha-OH-4-cholesten-3-one, and leukocyte LDL receptor and hydroxymethylglutaryl coenzyme A reductase mRNA were determined after each treatment period. Atorvastatin (10 mg/d) reduced LDL-C by an overall mean of 32.5%. Above-average responders (LDL-C -39.5%) had higher basal MVA levels (34.4+/-6.1 micromol/L) than did below-average responders (LDL-C -23.6%, P<0.02; basal MVA 26.3+/-6.1 micromol/L, P<0.01). Fewer good responders compared with the poor responders had an apolipoprotein E4 allele (3 of 11 versus 6 of 8, respectively; P<0.05). There were no baseline differences between them in 7-alpha-OH-4-cholesten-3-one, hydroxymethylglutaryl coenzyme A reductase mRNA, or LDL receptor mRNA, but the latter increased in the good responders on combination therapy (P<0.05). Severe mutations were not more common in poor than in good responders. We conclude that poor responders to statins have a low basal rate of cholesterol synthesis that may be secondary to a genetically determined increase in cholesterol absorption, possibly mediated by apolipoprotein E4. If so, statin responsiveness could be enhanced by reducing dietary cholesterol intake or inhibiting absorption.  (+info)

Growth hormone induces low-density lipoprotein clearance but not bile acid synthesis in humans. (5/41)

OBJECTIVE: Growth hormone (GH) induces hepatic low-density lipoprotein (LDL) receptors and lowers plasma cholesterol. We characterized the influence of GH treatment on plasma LDL clearance in normal humans and investigated the relative role of LDL receptor (LDLR) activity and stimulation of bile acid synthesis in subjects with different LDLR expression. METHODS AND RESULTS: Plasma clearance of autologous 125I-LDL was measured before and during 3 weeks of treatment with GH (0.1 IU/kg per day) in 9 healthy young males. Plasma LDL cholesterol was reduced by 13% and the fractional catabolic rate of LDL increased by 27%. More marked changes were seen in a patient with hypopituitarism substituted with GH (0.07 IU/kg per day) for 3 months. In a second study, GH dose-dependently reduced LDL cholesterol and increased Lp(a) levels in 3 groups of males: younger and elderly healthy subjects and heterozygous familial hypercholesterolemia (FH). No effect on bile acid synthesis measured by the plasma marker 7alpha-hydroxy-4-cholesten-3-one was observed. In an LDLR-deficient FH homozygote, LDL cholesterol was not affected by GH. CONCLUSIONS: GH treatment reduces plasma LDL cholesterol by inducing LDL clearance. In humans, LDLR expression is a prerequisite for this effect, whereas it is not related to stimulation of bile acid synthesis.  (+info)

Hepatic mRNA levels for the LDL receptor and HMG-CoA reductase show coordinate regulation in vivo. (6/41)

A sensitive solution hybridization assay using autologous cRNA probes was developed with the aim to study the simultaneous regulation of hepatic mRNA levels, on a quantitative basis, for the LDL receptor (LDLr), HMG-CoA reductase, and cholesterol 7 alpha-hydroxylase (Cho-7-hx) in C57BL/6J mice. With the purpose to suppress and stimulate transcript levels respectively, animals received established high fat diets, cholesterol-enriched diets, and a diet supplemented with mevinolin and colestipol. One hundred nineteen animals were investigated in six separate experiments. In spite of an eightfold increase in hepatic cholesterol induced by a high fat diet, the LDLr and the HMG-CoA reductase mRNA levels were only reduced to 60-70% and 25-50% of control values, respectively. When the data from all animals were analyzed, a strong positive correlation was obtained between the mRNA levels for the LDLr and HMG-CoA reductase (r = 0.79, P less than 0.001). A significant relation remained when control animals only were analyzed (n = 42, r = 0.59, P less than 0.001). Cho-7-hx mRNA showed a regulatory pattern that differed from that of the LDLr and HMG-CoA reductase; feeding cholesterol at 1.7% and 5% but not at 0.4% elevated the mRNA levels for Cho-7-hx while the LDLr and HMG-CoA reductase mRNA levels were maximally suppressed already at 0.4% of dietary cholesterol. The results show that the mRNA levels for the LDLr and HMG-CoA reductase are regulated in parallel in the liver in vivo during various metabolic perturbations as well as at normal physiologic conditions.(ABSTRACT TRUNCATED AT 250 WORDS)  (+info)

Comparison of computer- and human-derived coronary angiographic end-point measures for controlled therapy trials. (7/41)

The Cholesterol Lowering Atherosclerosis Study, a randomized angiographic clinical trial, demonstrated the beneficial effect of niacin/colestipol plus diet therapy on coronary atherosclerosis. Outcome was determined by panel-based estimates (viewed in both still and cine modes) of percent stenosis severity and change in native artery and bypass graft lesions. Computer-based quantitative coronary angiography (QCA) was also used to measure lesion and bypass graft stenosis severity and change in individual frames closely matched in orientation, opacification, and cardiac phase. Both methods jointly evaluated 350 nonoccluded lesions. The correlation between QCA and panel estimates of lesion size was 0.70 (p less than 0.0001) and for change in lesion size was 0.28 (p = 0.002). Agreement between the two methods in classifying lesion changes (i.e., regression, unchanged, or progression) occurred for 60% (210 of 350) of the lesions kappa +/- SEM = 0.20 +/- 0.05, p less than 0.001). The panel identified 442 nonoccluded lesions for which QCA stenosis measurements could not be obtained. Lesions not measurable by QCA included those with stenosis greater than 85% that could not be reliably edge tracked, segments with diffuse or ecstatic disease that had no reliable reference diameter, and segments for which matched frames could not be located. Seventy-nine lesions, the majority between 21% and 40% stenosis, were identified and measured by QCA but were not identified by the panel. This comparison study demonstrates the need to consider available angiographic measurement methods in relation to the goals of their use.  (+info)

Liver X receptor alpha interferes with SREBP1c-mediated Abcd2 expression. Novel cross-talk in gene regulation. (8/41)

The peroxisomal ATP binding cassette (ABC) transporter adrenoleukodystrophy-related protein, encoded by ABCD2, displays functional redundancy with the X-linked adrenoleukodystrophy-associated protein, making ABCD2 up-regulation of therapeutic value. Cholesterol lowering activates human ABCD2 in cultured cells. To investigate in vivo regulation by sterols, we first characterized a sterol regulatory element (SRE) in the murine Abcd2 promoter that is directly bound by SRE-binding proteins (SREBPs). Intriguingly, this element overlaps with a direct repeat 4, which serves as binding site for liver X receptor (LXR)/retinoid X receptor heterodimers, suggesting novel cross-talk between SREBP and LXR/retinoid X receptor in gene regulation. Using fasting-refeeding and cholesterol loading, SREBP accessibility to the SRE/direct repeat 4 was tested. Results suggest that adipose Abcd2 is induced by SREBP1c, whereas hepatic Abcd2 expression is down-regulated by concurrent activation of LXRalpha and SREBP1c. In cell culture, SREBP1c-mediated Abcd2 induction is counteracted by ligand-activated LXRalpha. Finally, hepatic Abcd2 expression in LXRalpha,beta-deficient mice is inducible to levels vastly exceeding wild type. Together, we identify LXRalpha as negative modulator of Abcd2, acting through a novel regulatory mechanism involving overlapping SREBP and LXRalpha binding sites.  (+info)

COLESTID® TABLETS (micronized colestipol HCl tablets) Warnings and Precautions | Pfizer Medical Information - NigeriaCOLESTID® TABLETS (micronized colestipol HCl tablets) Warnings and Precautions | Pfizer Medical Information - Nigeria
Since colestipol hydrochloride is an anion exchange resin, it may have a strong affinity for anions other than the bile acids. In vitro studies have indicated that colestipol hydrochloride binds a number of drugs. Therefore, COLESTID Tablets may delay or reduce the absorption of concomitant oral medication. The interval between the administration of COLESTID Tablets and any other medication should be as long as possible. Patients should take other drugs at least one hour before or four hours after COLESTID Tablets to avoid impeding their absorption.. Repeated doses of colestipol hydrochloride given prior to a single dose of propranolol in human trials have been reported to decrease propranolol absorption. However, in a follow-up study in normal subjects, single-dose administration of colestipol hydrochloride and propranolol and twice-a-day administration for 5 days of both agents did not affect the extent of propranolol absorption, but had a small yet statistically significant effect on its rate ...
https://www.pfizermedicalinformationng.com/en-us/colestid-tablets/warnings
Effects of combined therapy with lovastatin and colestipol in heterozygous familial hypercholesterolemia. Effects on kinetics...
Dive into the research topics of Effects of combined therapy with lovastatin and colestipol in heterozygous familial hypercholesterolemia. Effects on kinetics of apolipoprotein B. Together they form a unique fingerprint. ...
https://utsouthwestern.pure.elsevier.com/en/publications/effects-of-combined-therapy-with-lovastatin-and-colestipol-in-het/fingerprints/?sortBy=alphabetically
1990 - Niacin and lovastatin were more efficient than gemfibrozil, probucol, colestipol, or cholestyramine for...1990 - Niacin and lovastatin were more efficient than gemfibrozil, probucol, colestipol, or cholestyramine for...
The treatment of high blood cholesterol is entering a new phase. As in the treatment of hypertension, several drugs with different modes of action are now available, with more under study, kindling debates about their relative benefits and costs. The rationale and evidence for appropriate treatment of patients is becoming clearer, with the National Cholesterol Education Program guidelines leading the way. We are learning to decide which patients to treat with drug therapy and why, and we should all by now be familiar with how to use the available drugs. This study looks at drug treatment from a societal perspective by analyzing the cost-effectiveness of lowering cholesterol in an attempt to help the physician include costs in decisions about treatment. To simplify this, the authors used an LDL-HDL index to estimate the reduction in coronary heart disease morbidity and mortality for each cholesterol-lowering agent, based on the findings from the Lipid Research Clinics Primary Prevention Trial, ...
http://acpjc.org/Content/114/3/issue/ACPJC-1991-114-3-093.htm
JoVE Search Results: Colestipol
Atpase, ATP, Potassium, Sodium, Water, Membrane, Plasma, Atpases, Plasma Membrane, Kinase, Light, Epithelial Cells, and Membrane Proteins
http://labindex.jove.com/group/Colestipol
COLESTID® GRANULES (colestipol HCl for oral suspension) Adverse Reactions | Pfizer Información Médica - Costa RicaCOLESTID® GRANULES (colestipol HCl for oral suspension) Adverse Reactions | Pfizer Información Médica - Costa Rica
The most common adverse reactions are confined to the gastrointestinal tract. To achieve minimal GI disturbance with an optimal LDL-cholesterol lowering effect, a gradual increase of dosage starting with one dose/day is recommended. Constipation is the major single complaint and at times is severe. Most instances of constipation are mild, transient, and controlled with standard treatment. Increased fluid intake and inclusion of additional dietary fiber should be the first step; a stool softener may be added if needed. Some patients require decreased dosage or discontinuation of therapy. Hemorrhoids may be aggravated.. Other, less frequent gastrointestinal complaints consist of abdominal discomfort (abdominal pain and cramping), intestinal gas, (bloating and flatulence), indigestion and heartburn, diarrhea and loose stools, and nausea and vomiting. Bleeding hemorrhoids and blood in the stool have been infrequently reported. Peptic ulceration, cholecystitis, and cholelithiasis have been rarely ...
https://www.pfizermedicalinformation.co.cr/en-us/colestid-granules/adverse-reactions
COLESTID® TABLETS (micronized colestipol HCl tablets) Adverse Reactions | Pfizer Medical Information - NigeriaCOLESTID® TABLETS (micronized colestipol HCl tablets) Adverse Reactions | Pfizer Medical Information - Nigeria
The most common adverse reactions are confined to the gastrointestinal tract. To achieve minimal GI disturbance with an optimal LDL-C lowering effect, a gradual increase of dosage starting with 2 grams, once or twice daily is recommended. Constipation is the major single complaint and at times is severe. Most instances of constipation are mild, transient, and controlled with standard treatment. Increased fluid intake and inclusion of additional dietary fiber should be the first step; a stool softener may be added if needed. Some patients require decreased dosage or discontinuation of therapy. Hemorrhoids may be aggravated.. Other, less frequent gastrointestinal complaints consist of abdominal discomfort (abdominal pain and cramping), intestinal gas (bloating and flatulence), indigestion and heartburn, diarrhea and loose stools, and nausea and vomiting. Bleeding hemorrhoids and blood in the stool have been infrequently reported. Peptic ulceration, cholecystitis, and cholelithiasis have been ...
https://www.pfizermedicalinformationng.com/en-us/colestid-tablets/adverse-reactions
Drugs - colestipol - usage, dosage, interactions, images - AARP
Take this medicine by mouth with a full glass of water. Follow the directions on the prescription label. Tablets must be taken one at a time and swallowed whole. Do not cut, crush or chew. Take other medicines at least 1 hour before or 4 hours after this medicine. Take your doses at regular intervals. Do not take your medicine more often than directed ...
http://healthtools.aarp.org/goldcontent/colestipol
Evaluation of human panelists in assessing coronary atherosclerosis. | Arteriosclerosis, Thrombosis, and Vascular BiologyEvaluation of human panelists in assessing coronary atherosclerosis. | Arteriosclerosis, Thrombosis, and Vascular Biology
The Cholesterol Lowering Atherosclerosis Study, a randomized, angiographic clinical trial, has demonstrated the beneficial effect of niacin/colestipol therapy on coronary and femoral atherosclerosis. The primary outcome was a panel-determined consensus score evaluating global coronary changes determined angiographically at 2 years. This article presents an evaluation of interreader agreement in independently assessing the status of native coronary arteries and overall coronary condition. Parameters include 1) identification of the presence of lesions and lesion changes; 2) estimation of lesion severity (percent stenosis) and amount of change in lesion severity; and 3) global assessment of change in coronary status. Readers independently agreed on 1) presence of lesions (82%) and change in lesions (51%); 2) percent stenosis +/- 10% (76%) and change in stenosis +/- 10% (81%); and 3) global assessment of change in coronary status within one step (96%). Results of these analyses may be useful in ...
http://atvb.ahajournals.org/content/11/2/385
Familial hypercholesterolemia - WikipediaFamilial hypercholesterolemia - Wikipedia
FH is usually treated with statins.[7] Statins act by inhibiting the enzyme hydroxymethylglutaryl CoA reductase (HMG-CoA-reductase) in the liver. In response, the liver produces more LDL receptors, which remove circulating LDL from the blood. Statins effectively lower cholesterol and LDL levels, although sometimes add-on therapy with other drugs is required, such as bile acid sequestrants (cholestyramine or colestipol), nicotinic acid preparations or fibrates.[2] Control of other risk factors for cardiovascular disease is required, as risk remains somewhat elevated even when cholesterol levels are controlled. Professional guidelines recommend that the decision to treat a person with FH with statins should not be based on the usual risk prediction tools (such as those derived from the Framingham Heart Study), as they are likely to underestimate the risk of cardiovascular disease; unlike the rest of the population, FH have had high levels of cholesterol since birth, probably increasing their ...
https://en.m.wikipedia.org/wiki/Familial_hypercholesterolemia
Alzforum: AlzRisk Paper DetailAlzforum: AlzRisk Paper Detail
GHC patients receive prescriptions through the GHC pharmacy at no or nominal cost. The GHC pharmacy database was established in January 1977. Its data files contain information on drug, dosage, quantity dispensed, prescription date, and instructions. Use of statin medications (simvastatin, lovastatin, pravastatin, and atorvastatin) and other lipid-lowering agents (LLAs), including niacin,cholestyramine, colestipol, gemfibrozil, and clofibrate, was defined as at least three filled prescriptions for statins or LLAs of 15 tablets or more. Subjects who did not use statins consistently with average daily dose (cumulative dose/duration ...
http://alzrisk.org/paper.aspx?rfid=18&refrowid=758
Efficacy of Two Algae Formulations on Lipid Metabolism, Inflammation and Oxidative Stress Status in Individuals With Psoriasis ...Efficacy of Two Algae Formulations on Lipid Metabolism, Inflammation and Oxidative Stress Status in Individuals With Psoriasis ...
subjects on anticoagulant therapy (such as warfarin), taking medications and/or natural health products known to affect lipid metabolism (cholestyramine, colestipol, niacin, clofibrate, gemfibrozil, probucol, HMG CoA reductase inhibitors, high dose dietary supplements or fish oil capsules (,4g/day), guggul, lecithin, evening primrose oil within the last six month period will be excluded. In addition, subjects will not be allowed to consume any of these medications during the ...
https://clinicaltrials.gov/ct2/show/NCT01045395?term=TNFRSF1B&rank=5
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PatientsLikeMe | Colestipol report for patients like youPatientsLikeMe | Colestipol report for patients like you
Colestipol: Find the most comprehensive real-world treatment information on Colestipol at PatientsLikeMe. 15 patients with fibromyalgia, multiple sclerosis, major depressive disorder, generalized anxiety disorder, diabetes type 2, post-traumatic stress disorder, systemic lupus erythematosus, bipolar disorder, Parkinsons disease, panic disorder, rheumatoid arthritis, high blood pressure (hypertension), myalgic encephalomyelitis/chronic fatigue syndrome, persistent depressive disorder (dysthymia), amyotrophic lateral sclerosis, epilepsy, migraine, hypothyroidism, osteoarthritis, traumatic brain injury, bipolar II disorder, attention deficit/hyperactivity disorder, asthma, social anxiety disorder, high cholesterol (hypercholesterolemia), irritable bowel syndrome, idiopathic pulmonary fibrosis, gastroesophageal reflux disease, bipolar I disorder or psoriasis currently take Colestipol.
https://www.patientslikeme.com/treatments/show/2339-colestipol-suspension-side-effects-and-efficacy
Colestipol | YourCareEverywhereColestipol | YourCareEverywhere
This medicine is mixed into a liquid and taken by mouth. DO NOT take this medicine in the dry form. Follow the directions on the prescription label. Add the granules to 3 or more ounces of water, milk, fruit juice, pulpy fruit juice, fluid soup, or other liquid. Mix well and drink all the liquid (it will not dissolve). Rinse the glass with some additional liquid and swallow to make sure you take the full dose. If you use a carbonated drink, use a larger glass as the mixture will foam. Take other medicines at least 1 hour before or 4 hours after this medicine. Take your medicine at regular intervals. Do not take it more often than directed.. Talk to your pediatrician regarding the use of this medicine in children. Special care may be needed.. Overdosage: If you think you have taken too much of this medicine contact a poison control center or emergency room at once.. NOTE: This medicine is only for you. Do not share this medicine with others.. ...
https://www.yourcareeverywhere.com/article/krames/en/article/health-research/drugs-and-supplements/drug-search/colestipol.html
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Do not use Generic Ziac if you are allergic to Generic Ziac components, to sulfa drugs or to any beta-blocker medication, such as atenolol (Tenormin), bisoprolol (Zebeta), labetalol (Normodyne, Trandate), nadolol (Corgard), propranolol (Inderal, InnoPran), sotalol (Betapace), timolol (Blocadren), and others.. Do not use Generic Ziac if youre pregnant or you plan to have a baby. Do not take it in case you are a nursing mother.. Do not use Generic Ziac if you are unable to urinate.. Do not use Generic Ziac if you have severe or uncontrolled heart failure, a heart condition called sick sinus syndrome or AV block or slow heartbeats.. Be careful with Generic Ziac if you have congestive heart failure, circulation problems, kidney or liver disease, cirrhosis, glaucoma, asthma, bronchospastic lung disease, a thyroid disorder, lupus, gout, diabetes, or a penicillin allergy.. Be careful with Generic Ziac if you take insulin or oral diabetes medication; colestipol (Colestid) or cholestyramine ...
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Intensive Lipid-Lowering Therapy for Patients With Acute Coronary Syndrome - No Study Results Posted - ClinicalTrials.govIntensive Lipid-Lowering Therapy for Patients With Acute Coronary Syndrome - No Study Results Posted - ClinicalTrials.gov
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Plasma Squalene as an Index of Cholesterol Synthesis | Clinical Science | Portland PressPlasma Squalene as an Index of Cholesterol Synthesis | Clinical Science | Portland Press
1. Seven subjects were studied before and after the rate of cholesterol synthesis was altered with drugs or dietary cholesterol.. 2. The rate at which plasma free cholesterol was formed from squalene during constant infusions of radioactive mevalonate increased with colestipol treatment, decreased with clofibrate treatment and decreased when cholesterol was added to the diet. The plasma squalene concentration showed corresponding changes, confirming that its measurement may qualitatively define changes in cholesterol synthesis.. 3. The mean plasma squalene concentration in seven hypertriglyceridaemic, slightly overweight subjects was significantly higher than in six hypercholesterolaemic subjects, which is consistent with other evidence for increased cholesterol synthesis in hypertriglyceridaemia. ...
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Niacor Information - Drugs and Treatments - MedHelpNiacor Information - Drugs and Treatments - MedHelp
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Sunlight and Vitamin D improve mood | DrDobbin NutritionSunlight and Vitamin D improve mood | DrDobbin Nutrition
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Differences in SequestrantsDifferences in Sequestrants
In a final effort to get the yellow out of my water, I tried a third brand of sequestrant/chelating agent, Sequa-Sol It worked. The water is the best looking it has been to date. The previous two were Metal Free and Sea Klear Metal Klear. The MSDS on this new one says the active ingredient is Sodium Citrate. It is listed as a chelating agent where the previous two used Acrylamide-Acrylic Acid Copolymer, listed as a sequestrant. I cant find much that explains the difference, at least as
http://www.troublefreepool.com/threads/6923-Differences-in-Sequestrants
Bile acid sequestrant - WikipediaBile acid sequestrant - Wikipedia
The bile acid sequestrants are a group of resins used to bind certain components of bile in the gastrointestinal tract. They disrupt the enterohepatic circulation of bile acids by combining with bile constituents and preventing their reabsorption from the gut. In general, they are classified as hypolipidemic agents, although they may be used for purposes other than lowering cholesterol. They are used in the treatment of chronic diarrhea due to bile acid malabsorption. Bile acid sequestrants are polymeric compounds that serve as ion-exchange resins. Bile acid sequestrants exchange anions such as chloride ions for bile acids. By doing so, they bind bile acids and sequester them from the enterohepatic circulation. The liver then produces more bile acids to replace those that have been lost. Because the body uses cholesterol to make bile acids, this reduces the amount of LDL cholesterol circulating in the blood. Bile acid sequestrants are large polymeric structures, and they are not significantly ...
https://en.wikipedia.org/wiki/Bile_acid_sequestrant
Bile Acid Sequestrants and the Treatment of Type 2 Diabetes Mellitus | SpringerLinkBile Acid Sequestrants and the Treatment of Type 2 Diabetes Mellitus | SpringerLink
Bile acids promote bile formation and facilitate dietary lipid absorption. Animal and human studies showing disturbed bile acid metabolism in diabetes mellitus suggest a link between bile acids and glucose control. Bile acids are activating ligands of the farnesoid X receptor (FXR), a nuclear receptor with an established role in bile acid and lipid metabolism. Evidence suggests a role for FXR also in maintenance of glucose homeostasis. Animal and human studies employing bile acid sequestrants (bile acid binding agents), which interrupt the enterohepatic circulation of bile acids and effectively reduce plasma cholesterol, support a link between bile acid and glucose metabolism. In lipid-lowering trials, bile acid sequestrants, such as colesevelam hydrochloride, colestyramine (cholestyramine) and colestilan (colestimide), have also been shown to lower plasma glucose and glycosylated haemoglobin levels, suggesting the utility of these agents as a potential therapy for type 2 diabetes. In this ...
https://link.springer.com/article/10.2165%2F00003495-200767100-00001
Bile Acid Sequestrant Drugs | Grand Strand HealthBile Acid Sequestrant Drugs | Grand Strand Health
Learn more about Bile Acid Sequestrant Drugs at Grand Strand Medical Center Many Nutrients - Supplementation Likely Helpful The bile acid sequestrant...
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Which medications in the drug class Bile Acid Sequestrants are used in the treatment of Crohn Disease?Which medications in the drug class Bile Acid Sequestrants are used in the treatment of Crohn Disease?
Bile Acid SequestrantsPatients with terminal ileal disease may not absorb bile acids normally, which can lead to secretory diarrhea in the colon. These patients may benefit from bile acid sequestrants... more
https://www.medscape.com/answers/172940-18379/937574-overview
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This drug is not recommended for use with: dofetilide. Ask your doctor or pharmacist for more details. Inform your doctor about all the medicines you may use (both prescription and nonprescription), especially if you take: lithium, digoxin, oral drugs used for diabetes, aspirin, NSAIDs (e.g., ibuprofen, naproxen), fluconazole. If you take colestipol or cholestyramine for high cholesterol, take the diuretic 1 hour before or 4 hours after because of decreased absorption into the bloodstream. Avoid any drugs that increase your heart rate or make you excited like decongestants because it may counter-act your blood pressure medicine. Decongestants are commonly found in over the counter cough-and-cold products. Ask your pharmacist if you are uncertain about decongestants in over-the-counter products. Do not start or stop any medicine without doctor or pharmacist approval. ...
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Therapy Drug-drug interactions displayed by pressing [f3] while in the list of drugs will now display details of each interaction. The listings for drug toxicity and interactions have been extensively updated and expanded Drugs added Levofloxacin Pharmacological profile Ampicillin-sulbactam, Cefotetan, Doxycycline, Meningococcal vaccine, Ofloxacin, Pyrimethamine / sulfadoxine Susceptibility database Eubacterium lentum Testing criteria Levofloxaxin Interactions database Atorvastatin, Colestipol, Insulin, Molindone, Pirenzepine, Zalcitabine Microbiology Note: A variety of fermentation and other reactions for gram-positive bacilli and non-fermentative gram-negative bacilli may require three or more days. The GIDEON data base allows for delayed positivity in such cases. Therefore, do not indicate a negative response for such organisms until at least three days have passed (see individual notes [F2] in the identification module). New taxa added Achromobacter group B, Achromobacter group E, ...
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Reduction of Serum Cholesterol Levels Alters Lesional Composition of Atherosclerotic Plaques | Arteriosclerosis, Thrombosis,...Reduction of Serum Cholesterol Levels Alters Lesional Composition of Atherosclerotic Plaques | Arteriosclerosis, Thrombosis,...
Many studies have demonstrated that progression of atherosclerosis can be suppressed by reduction of serum cholesterol levels. In these studies, however, histopathological changes of the atherosclerotic lesions have not been sufficiently examined. Results of our study show that a reduction of serum cholesterol levels due to pravastatin treatment altered the lesional composition of atherosclerotic plaques in WHHL rabbits.. In the present study we showed that a long-term reduction of serum cholesterol levels led to the following histopathological changes in the atherosclerotic plaques: (1) a decrease in the area of macrophages plus extracellular lipid deposits, (2) an increase in collagen area, (3) a suppression of the decrease in smooth muscle cell area with lesion progress, and (4) an increase in the ratio of collagen area to the area of extracellular lipid deposits (Tables 3⇑ and 5⇑).. In recent human studies, several groups reported on acute clinical coronary events. Becker and coworkers ...
http://atvb.ahajournals.org/content/15/11/1938
Risk factors for clinical coronary heart disease in systemic lupus erythematosus: the lupus and atherosclerosis evaluation of...Risk factors for clinical coronary heart disease in systemic lupus erythematosus: the lupus and atherosclerosis evaluation of...
Haque, Sahena, Gordon, Caroline, Isenberg, David, Rahman, Anisur, Lanyon, Peter, Bell, Aubrey, Emery, Paul, McHugh, Neil, Teh, Lee Suan, Scott, David G. I., Akil, Mohamed, Naz, Sophia, Andrews, Jacqueline, Griffiths, Bridget, Harris, Helen, Youseff, Hazem, McLaren, John, Toescu, Veronica, Devakumar, Vinodh, Teir, Jamal and Bruce, Ian N. (2010) Risk factors for clinical coronary heart disease in systemic lupus erythematosus: the lupus and atherosclerosis evaluation of risk (LASER) study. Journal of Rheumatology, 37 (2). pp. 322-329. Full text not available from this repository. (Request a copy ...
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One-Alpha Drops | Medicines.ieOne-Alpha Drops | Medicines.ie
In Section 4.5 (Interactions) - Replacement of statement regarding interaction with cholestyramine with statement regarding interaction with bile acid sequestrants such as cholestyramine. - In Section 4.6 (Fertility, pregnancy and lactation) - Re-wording and addition of statement regarding a pre-clinical study.. - In Section 4.8 (Undesirable effects) - Deletion of list of symptoms of hypercalcaemia. Deletion of frequency number definitions from tabulation.. - Minor clarifications and re-wordings in sections 2, 4.2, 4.4 and 6.6 ...
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Vytorin Alternatives
Vytorin alternatives often include statins or other cholesterol medicines. This eMedTV article lists various types of statins and other classes of cholesterol medications, such as bile acid sequestrants, fibrates, and niacin.
http://cholesterol.emedtv.com/vytorin/vytorin-alternatives.html
DMOZ - Health: Pharmacy: Drugs and Medications: Lipid-Lowering Agents
Drugs to reduce cholesterol and other lipids in blood. Classes of drugs used for this purpose include statins, bile acid sequestrants, nicotinic acid, and fibrates.
http://dmoztools.net/Health/Pharmacy/Drugs_and_Medications/Lipid-Lowering_Agents/
L-Methylfolate Drug Interactions
Some anti-seizure medicines and bile acid sequestrants can react negatively with L-methylfolate. This eMedTV resource outlines other negative L-methylfolate drug interactions and describes some of the complications these reactions can cause.
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Diagnostic scoring for familial hypercholesterolaemia in practice -ORCADiagnostic scoring for familial hypercholesterolaemia in practice -ORCA
Purpose of review: Diagnostic scoring for familial hypercholesterolaemia (FH) can be used either to screen for possible FH or guide the selection of patients for genetic (DNA) testing. We review the published diagnostic criteria and discuss the options for future development. Recent findings: Scoring systems have been developed internationally based on lipid values and various combinations of clinical signs and cardiovascular history. The predictive value varies according to the test population, be it lipid clinic referrals, general population, or relatives of patients with FH. Also, there is increasing recognition of genetic heterogeneity in FH so that criteria are of differing predictive value depending on the genetic variant of FH. Summary: These clinical scoring systems are increasingly used to guide selection of patients for FH genetic testing but no single approach has yet emerged as the system of choice. Further refinement of these scoring tools using more sophisticated calculators are ...
http://orca.cf.ac.uk/102866/
Lojuxta (lomitapide): a novel treatment for homozygous familial hypercholesterolaemiaLojuxta (lomitapide): a novel treatment for homozygous familial hypercholesterolaemia
Pharmacology and key clinical studies of Lojuxta (lomitapide), a selective inhibitor of microsomal transfer protein (MTP) for homozygous familial hypercholesterolaemia.
https://www.mims.co.uk/lojuxta-new-treatment-homozygous-familial-hypercholesterolaemia/cardiovascular-system/article/1221403
Familial hypercholesterolaemia - EndodiabesityFamilial hypercholesterolaemia - Endodiabesity
Are you in need of a quick refresher on FH? Then this review article in the BMJ is for you. It is, however, not without inaccuracies and you could do worse than look up NICE Clinical Guideline 71 for a more thorough reading of the subject.
https://endodiabesity.org/2012/06/10/familial-hypercholesterolaemia/
Familial hypercholesterolaemia: identification and management - DROFamilial hypercholesterolaemia: identification and management - DRO
Every reasonable effort has been made to ensure that permission has been obtained for items included in DRO. If you believe that your rights have been infringed by this repository, please contact [email protected] ...
http://dro.deakin.edu.au/view/DU:30109615
Familial hypercholesterolaemia | Sonic GeneticsFamilial hypercholesterolaemia | Sonic Genetics
Sonic Genetics provides word-class genetic testing through specialised genetic laboratories across Australia, the UK, Europe and the USA.
https://www.sonicgenetics.com.au/our-tests/all-tests/familial-hypercholesterolaemia/
Bile acid sequestrants for cholesterolBile acid sequestrants for cholesterol
Davidson DJ, Wilkinson MJ, Davidson MH. Combination therapy for dyslipidemia. In: Ballantyne CM, ed. Clinical Lipidology: A Companion to Braunwalds Heart Disease. 2nd ed. Philadelphia, PA: Elsevier Saunders; 2015:chap 27.. Genest J, Libby P. Lipoprotein disorders and cardiovascular disease. In: Zipes DP, Libby P, Bonow RO, Mann DL, Tomaselli GF, Braunwald E, eds. Braunwalds Heart Disease: A Textbook of Cardiovascular Medicine. 11th ed. Philadelphia, PA: Elsevier; 2019:chap 48.. Goldberg AC. Bile acid sequestrants. In: Ballantyne CM, ed. Clinical Lipidology: A Companion to Braunwalds Heart Disease. 2nd ed. Philadelphia, PA: Elsevier Saunders; 2015:chap 22.. Grundy SM, Stone NJ, Bailey AL, et al. 2018 AHA/ACC/AACVPR/AAPA/ABC/ACPM/ ADA/AGS/APhA/ASPC/NLA/PCNA guideline on the management of blood cholesterol: a report of the American College of Cardiology/American Heart Association Task Force on Clinical Practice Guidelines. J Am Coll Cardiol. 2019;73(24):e285-e350. PMID: 30423393 ...
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Familial hypercholesterolaemia<...
TY - JOUR. T1 - Familial hypercholesterolaemia. AU - Nair, Devaki R.. AU - Sharifi, Mahtab. AU - Al-Rasadi, Khalid. PY - 2014. Y1 - 2014. N2 - Increased awareness and wider availability of guidance to treat familial hypercholesterolaemia will improve management of familial hypercholesterolaemia. New therapies, if they become available after appropriate outcome studies, will reduce LDL-C levels in both homozygous familial hypercholesterolaemia and severe heterozygous familial hypercholesterolaemia, thus reducing the risk for premature CHD.. AB - Increased awareness and wider availability of guidance to treat familial hypercholesterolaemia will improve management of familial hypercholesterolaemia. New therapies, if they become available after appropriate outcome studies, will reduce LDL-C levels in both homozygous familial hypercholesterolaemia and severe heterozygous familial hypercholesterolaemia, thus reducing the risk for premature CHD.. KW - Familial hypercholesterolaemia. KW - Guidelines for ...
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South African Dyslipidaemia Guideline Consensus StatementSouth African Dyslipidaemia Guideline Consensus Statement
Bile acid sequestrants and nicotinic acid have cholesterollowering properties. They may occasionally be useful alone or in combination with statin therapy. However, their side-effects limit wider application.. 8.8 Treatment directed at other components of the lipid profile. Whereas low levels of HDL-C and high levels of TG are undoubtedly associated with a higher cardiovascular disease risk, no currently available treatment directed at reversing these changes has been shown to significantly benefit cardiovascular outcome.. A high triglyceride level, particularly if ,10 mmol/l, can result in acute pancreatitis and should be treated without delay.. 9. Special circumstances. 9.1 Metabolic syndrome. The European Guidelines recognise the importance of identifying patients with the metabolic syndrome, who are at increased risk of cardiovascular disease. The presence of the syndrome approximately doubles the risk of cardiovascular disease. Lifestyle changes, particularly reducing body weight and ...
http://www.scielo.org.za/scielo.php?script=sci_arttext&pid=S0256-95742012000300032&lng=en&nrm=iso&tlng=en
Familial hypercholesterolaemia: identification and management | Guidance and guidelines | NICEFamilial hypercholesterolaemia: identification and management | Guidance and guidelines | NICE
Evidence-based recommendations on identifying and managing familial hypercholesterolaemia (inherited high cholesterol) for adults and children
https://www.nice.org.uk/guidance/cg71/chapter/Update-information
Familial hypercholesterolaemia is underdiagnosed and undertreated in the general population - Danish National Research Database...Familial hypercholesterolaemia is underdiagnosed and undertreated in the general population - Danish National Research Database...
The first aim was to critically evaluate the extent to which familial hypercholesterolaemia (FH) is underdiagnosed and undertreated. The second aim was to provide guidance for screening and treatment of FH, in order to prevent coronary heart disease (CHD ...
http://www.forskningsdatabasen.dk/catalog/255851344
Molecular analysis of patients with familial hypercholesterolaemia (FH): evaluation of two high-throughput mutation analysis...Molecular analysis of patients with familial hypercholesterolaemia (FH): evaluation of two high-throughput mutation analysis...
UCL Discovery is UCLs open access repository, showcasing and providing access to UCL research outputs from all UCL disciplines.
http://discovery.ucl.ac.uk/97330/
ColestipolColestipol
Drugs.com: Colestipol Hydrochloride "colestipol (Colestid)". MedicineNet. Mutschler E, Schäfer-Korting M (2001). ... ISBN 3-8047-1763-2. Clinical Pharmacology: Colestipol structure Beth Israel Deaconess Medical Center & Care Group: Colestipol ... Colestipol is contraindicated in hypertriglyceridemia (high level of triglycerides in the blood).[citation needed] Colestipol ... Like cholestyramine, colestipol works in the gut by trapping bile acids and preventing them from being reabsorbed. This leads ...
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Bile acid sequestrantBile acid sequestrant
Cholestyramine, colestipol and colesevelam have all been used. Doses may not need to be as high as those previously used for ... Heel RC, Brogden RN, Pakes GE, Speight TM, Avery GS (March 1980). "Colestipol: a review of its pharmacological properties and ... Colestipol (Colestid, Colestipid) Colesevelam (Cholestagel in Europe, Welchol in the US, Lodalis in Canada) Wilcox C, Turner J ...
https://en.wikipedia.org/wiki/Bile_acid_sequestrant
Ion-exchange resinIon-exchange resin
Colestipol is a weakly basic ion-exchange resin and is used to treat hypercholesterolemia. Cholestyramine is a strongly basic ... Colestipol and cholestyramine are known as bile acid sequestrants. Ion-exchange resins are also used as excipients in ... Three ion-exchange resins, sodium polystyrene sulfonate, colestipol, and cholestyramine, are used as active ingredients. Sodium ...
https://en.wikipedia.org/wiki/Ion-exchange_resin
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Poison Prevention Packaging Act of 1970Poison Prevention Packaging Act of 1970
Colestipol in powder form in packages containing no more than 5 grams of the drug. Conjugated estrogen tablets when dispensed ...
https://en.wikipedia.org/wiki/Poison_Prevention_Packaging_Act_of_1970
Primary biliary cholangitisPrimary biliary cholangitis
First-line treatment of pruritus consists of anion-exchange resins, such as cholestyramine, colestipol, or colesevalam. These ...
https://en.wikipedia.org/wiki/Primary_biliary_cholangitis
LevothyroxineLevothyroxine
... colestipol, and polystyrene sulfonate. Sevelamer with calcium carbonate may decrease the bioavailability of levothyroxine. ...
https://en.wikipedia.org/wiki/Levothyroxine
SitosterolemiaSitosterolemia
If dietary treatment alone is insufficient, bile acid-binding resins (e.g., cholestyramine, colestipol) could be considered. In ...
https://en.wikipedia.org/wiki/Sitosterolemia
DyslipidemiaDyslipidemia
Resins include cholestyramine, colestipol, and colesevalem, and they all decrease LDL-C while increasing HDL-C levels slightly ...
https://en.wikipedia.org/wiki/Dyslipidemia
HyperlipidemiaHyperlipidemia
Bile acid binding resins, such as colestipol, cholestyramine, and colesevelam, function by binding bile acids, increasing their ...
https://en.wikipedia.org/wiki/Hyperlipidemia
Postcholecystectomy syndromePostcholecystectomy syndrome
... colestipol or colesevelam, which may be better tolerated. Symptoms of postcholecystectomy syndrome may include: Dyspepsia, ...
https://en.wikipedia.org/wiki/Postcholecystectomy_syndrome
Hashimotos thyroiditisHashimoto's thyroiditis
Finally, cholestryamine, colestipol, aluminium hydroxide, ferrous sulphate, and sucralfate are all drugs that decrease T4 ...
https://en.wikipedia.org/wiki/Hashimoto's_thyroiditis
FenofibrateFenofibrate
... colestipol, etc.): If taken together, bile acid resins may bind to fenofibrate, resulting in a decrease in fenofibrate ...
https://en.wikipedia.org/wiki/Fenofibrate
Familial hypercholesterolemiaFamilial hypercholesterolemia
... cholestyramine or colestipol), nicotinic acid preparations or fibrates. Control of other risk factors for cardiovascular ...
https://en.wikipedia.org/wiki/Familial_hypercholesterolemia
List of MeSH codes (D25)List of MeSH codes (D25)
... colestipol MeSH D25.720.259 - cyanoacrylates MeSH D25.720.259.341 - enbucrilate MeSH D25.720.259.341.110 - bucrylate MeSH ...
https://en.wikipedia.org/wiki/List_of_MeSH_codes_(D25)
List of drugs: Cm-CoList of drugs: Cm-Co
... colestipol (INN) colestolone (INN) colestyramine (INN) colextran (INN) colfenamate (INN) colforsin (INN) colfosceril palmitate ...
https://en.wikipedia.org/wiki/List_of_drugs:_Cm–Co
List of MeSH codes (D02)List of MeSH codes (D02)
... colestipol MeSH D02.092.782.258 - diamines MeSH D02.092.782.258.174 - cadaverine MeSH D02.092.782.258.368 - ethylenediamines ...
https://en.wikipedia.org/wiki/List_of_MeSH_codes_(D02)
ATC code C10ATC code C10
C10AB08 Ciprofibrate C10AB09 Etofibrate C10AB10 Clofibride C10AB11 Choline fenofibrate C10AC01 Colestyramine C10AC02 Colestipol ...
https://en.wikipedia.org/wiki/ATC_code_C10
Colestipol: MedlinePlus Drug InformationColestipol: MedlinePlus Drug Information
Colestipol: learn about side effects, dosage, special precautions, and more on MedlinePlus ... Before taking colestipol,. *tell your doctor and pharmacist if you are allergic to colestipol, any other medications, or any of ... Continue to take colestipol even if you feel well. Do not stop taking colestipol without talking to your doctor. ... Take colestipol exactly as directed. Do not take more or less of it or take it more often than prescribed by your doctor. ...
https://medlineplus.gov/druginfo/meds/a682157.html
Colestipol - Health Library
Before taking colestipol, *tell your doctor and pharmacist if you are allergic to colestipol, any other medications, or any of ... Continue to take colestipol even if you feel well. Do not stop taking colestipol without talking to your doctor. ... Take colestipol exactly as directed. Do not take more or less of it or take it more often than prescribed by your doctor. ... Colestipol comes as tablets and granules to take by mouth. The tablets are usually taken once or twice daily. The granules are ...
https://healthlibrary.epnet.com/GetContent.aspx?token=99744adb-4d1c-491b-af5f-aafc5521ac7b&chunkiid=673897
colestipol hydrochloride | Tabers Medical Dictionarycolestipol hydrochloride | Taber's Medical Dictionary
colestipol hydrochloride answers are found in the Tabers Medical Dictionary powered by Unbound Medicine. Available for iPhone ... colestipol hydrochloride is a topic covered in the Tabers Medical Dictionary. To view the entire topic, please log in or ... "Colestipol Hydrochloride." Tabers Medical Dictionary, 24th ed., F.A. Davis Company, 2021. Tabers Online, www.tabers.com/ ... tabersonline/view/Tabers-Dictionary/734682/all/colestipol_hydrochloride. Colestipol hydrochloride. In: Venes DD, ed. Tabers ...
https://www.tabers.com/tabersonline/view/Tabers-Dictionary/734682/all/colestipol_hydrochloride?refer=true
Clinical trial: A Prospective, Open Label Comparison of Ezetimibe, Niacin, and Colestipol as Adjunct Therapy in Lipid ReductionClinical trial: A Prospective, Open Label Comparison of Ezetimibe, Niacin, and Colestipol as Adjunct Therapy in Lipid Reduction
... and Colestipol as Adjunct Therapy in Lipid Reduction ... or colestipol - Consumes more than 8oz. grapefruit juice daily ... Intervention: Niacin (Drug); Colestipol (Drug); Ezetimibe (Drug). Phase: Phase 4. Status: Completed. Sponsored by: Tuscaloosa ... ezetimibe, niacin, or colestipol Locations and Contacts. Tuscaloosa Research & Education Advancement Corporation, Tuscaloosa, ... A Prospective, Open Label Comparison of Ezetimibe, Niacin, and Colestipol as Adjunct Therapy in Lipid Reduction. Information ...
http://www.druglib.com/trial/76/NCT00203476.html
Buy Colestid (colestipol hcl) Online | Blink HealthBuy Colestid (colestipol hcl) Online | Blink Health
Colestipol is used along with a proper diet to lower cholesterol in the blood. ... Before taking colestipol, tell your doctor or pharmacist if you are allergic to it; or if you have any other allergies. This ... Colestipol is used along with a proper diet to lower cholesterol in the blood. Lowering cholesterol helps decrease the risk for ... Colestipol is used along with a proper diet to lower cholesterol in the blood. Lowering cholesterol helps decrease the risk for ...
https://www.blinkhealth.com/colestid
Crohn Disease Medication: 5-Aminosalicylic Acid Derivatives, Corticosteroids, Immunosuppressants, Monoclonal Antibodies, Alpha...Crohn Disease Medication: 5-Aminosalicylic Acid Derivatives, Corticosteroids, Immunosuppressants, Monoclonal Antibodies, Alpha...
Colestipol forms a soluble complex after binding to bile acid, increasing fecal loss of bile acid-bound low-density lipoprotein ... These patients may benefit from bile acid sequestrants such as cholestyramine (2-4 g) and colestipol (5 g 2 or 3 times daily ... These patients may benefit from bile acid sequestrants such as cholestyramine and colestipol. ...
https://www.medscape.com/answers/172940-15051/when-are-bile-acid-sequestrants-indicated-in-the-management-of-crohn-disease
Colestid (Colestipol Hydrochloride)Colestid (Colestipol Hydrochloride)
h1,COLESTID (COLESTIPOL),/h1, ,h2,Colestid Description,/h2, ,p,While cholesterol has many functions in your body, including ... COLESTID (COLESTIPOL). Colestid Description. While cholesterol has many functions in your body, including helping produce bile ... The active ingredient of Colestid is Colestipol, which is classed as a bile acid sequestrant drug. ...
https://discountdrugsfromcanada.com/colestid-colestipol-hydrochloride.html
Colestipol (Colestid) | Daviss Drug GuideColestipol (Colestid) | Davis's Drug Guide
Find information on Colestipol (Colestid) in Daviss Drug Guide including dosage, side effects, interactions, nursing ... "Colestipol." Daviss Drug Guide, 18th ed., F.A. Davis Company, 2023. Daviss Drug Guide - OLD - USE 2.0, www.drugguide.com/ddo/ ... view/Davis-Drug-Guide/51172/all/colestipol. Vallerand AHA, Sanoski CAC, Quiring CC. Colestipol. Daviss Drug Guide. F.A. Davis ... colestipol is a topic covered in the Daviss Drug Guide. To view the entire topic, please log in or purchase a subscription. ...
https://www.drugguide.com/ddo/view/Davis-Drug-Guide/51172/all/colestipol?q=amiodarone
Generic Colestid Availability - Drugs.comGeneric Colestid Availability - Drugs.com
colestipol hydrochloride granule;oral. * Manufacturer: IMPAX LABS. Approval date: May 2, 2006. Strength(s): 5GM/SCOOPFUL [AB], ... COLESTID (colestipol hydrochloride - tablet;oral). * Manufacturer: PFIZER. Approval date: July 19, 1994. Strength(s): 1GM [RLD] ... colestipol hydrochloride tablet;oral. * Manufacturer: IMPAX LABS. Approval date: October 24, 2006. Strength(s): 1GM [AB] ... COLESTID (colestipol hydrochloride - granule;oral). * Manufacturer: PFIZER. Approval date: September 22, 1995. Strength(s): 5GM ...
https://www.drugs.com/availability/generic-colestid.html
Colestipol - For All Medical Treatment Options Explained, Visit CureCrowdColestipol - For All Medical Treatment Options Explained, Visit CureCrowd
Colestipol. Also Known As: Colestipol, Colestid, Cholestabyl. Colestipol is a bile acid sequestrant. It works in the gut ... Complete a survey on Colestipol to help the CureCrowd community. If you have tried to treat this ailment, please complete the ...
https://www.curecrowd.com/treatments/colestid
Cholesterol Drugs: What to Expect With Heart MedicationCholesterol Drugs: What to Expect With Heart Medication
Prescribed for: Colestipol (Colestid) is used for the treatment of high cholesterol in conjunction with dietary control; for ... colestipol (Colestid). Drug Class: Bile acid sequestrants. Prescription: Yes. Generic: Yes. Preparations: Tablet of 1 gm. ... Side effects: Side effects of colestipol (Colestid) include constipation, stomach upset, heartburn, indigestion, belching, gas ...
https://www.emedicinehealth.com/slideshow_cholesterol_drugs/article_em.htm
ATSDR - Oak Ridge Reservation - ORRHES Meeting MinutesATSDR - Oak Ridge Reservation - ORRHES Meeting Minutes
Cholestyramine, colestipol. Explanations for rise of TSH in patient on same dose of T4:. *Accelerated metabolism ...
https://www.atsdr.cdc.gov/sites/oakridge/orr/m12_3_02p3.html
DailyMed - CLINDAMYCIN PHOSPHATE lotionDailyMed - CLINDAMYCIN PHOSPHATE lotion
Cholestyramine or colestipol resins bind vancomycin in vitro. If both a resin and vancomycin are to be administered ...
https://dailymed.nlm.nih.gov/dailymed/lookup.cfm?setid=f8c7339a-5180-4c6a-99bf-8a7e4d94f2d2
Crohn Disease: Practice Essentials, Background, PathophysiologyCrohn Disease: Practice Essentials, Background, Pathophysiology
Bile acid sequestrants (eg, cholestyramine, colestipol). * Anticholinergic agents (eg, dicyclomine, hyoscyamine, propantheline) ...
http://emedicine.medscape.com/article/172940
Mycophenolate Mofetil (Oral Route) Before Using - Mayo ClinicMycophenolate Mofetil (Oral Route) Before Using - Mayo Clinic
Although certain medicines should not be used together at all, in other cases two different medicines may be used together even if an interaction might occur. In these cases, your doctor may want to change the dose, or other precautions may be necessary. When you are taking this medicine, it is especially important that your healthcare professional know if you are taking any of the medicines listed below. The following interactions have been selected on the basis of their potential significance and are not necessarily all-inclusive.. Using this medicine with any of the following medicines is usually not recommended, but may be required in some cases. If both medicines are prescribed together, your doctor may change the dose or how often you use one or both of the medicines.. ...
https://www.mayoclinic.org/drugs-supplements/mycophenolate-mofetil-oral-route/before-using/drg-20073191
DailyMed - LEVOTHYROXINE SODIUM tabletDailyMed - LEVOTHYROXINE SODIUM tablet
Colestipol Ion Exchange Resins -Kayexalate -Sevelamer Bile acid sequestrants and ion exchange resins are known to decrease ...
https://dailymed.nlm.nih.gov/dailymed/drugInfo.cfm?setid=75d77bc7-151a-74b8-e053-2991aa0a9278
Listing of Drugs Currently Regulated as New Drugs (The New Drugs List) - Canada.caListing of Drugs Currently Regulated as New Drugs (The New Drugs List) - Canada.ca
RxAssist - RXOUTREACH Patient Assist ProgramRxAssist - RXOUTREACH Patient Assist Program
A resource to help physicians, advocates, and patients access free medications through pharmaceutical company patient assistance programs.
https://www.rxassist.org/pap-info/company-detail?CmpId=132
Pro-Indapamide - Uses, Side Effects, Interactions - MedBroadcast.comPro-Indapamide - Uses, Side Effects, Interactions - MedBroadcast.com
colestipol. *inhaled corticosteroids (e.g., budesonide, ciclesonide, fluticasone). *oral corticosteroids (e.g., dexamethasone, ...
https://medbroadcast.com/drug/getdrug/pro-indapamide
Triquilar - Uses, Side Effects, Interactions - MedBroadcast.comTriquilar - Uses, Side Effects, Interactions - MedBroadcast.com
colestipol. *conivaptan. *corticosteroids (e.g., dexamethasone, prednisone, hydrocortisone). *cyclosporine. *cyclobenzaprine. * ...
https://www.medbroadcast.com/drug/getdrug/triquilar
Our Pfizer Products Hold the Cure | PfizerOur Pfizer Products Hold the Cure | Pfizer
In this section find an overview of our Pfizer Products and search for information on our most popular Pfizer products. Our products are the results of 1500 scientists overseeing more than 500,000 lab tests and over 36 clinical trials before the first prescription. Our Pfizer products hold the cure.
https://www.pfizer.com/products?letter=V
How Cholesterol Works | HowStuffWorksHow Cholesterol Works | HowStuffWorks
Colestipol (Colestid) The B vitamin Niacin, in high doses, can lower triglycerides and LDL levels and increase HDL levels. ...
https://health.howstuffworks.com/diseases-conditions/cardiovascular/cholesterol/cholesterol.htm
Dynacin, Minocin (minocycline) dosing, indications, interactions, adverse effects, and moreDynacin, Minocin (minocycline) dosing, indications, interactions, adverse effects, and more
colestipol. Minor (1)colestipol decreases levels of minocycline by inhibition of GI absorption. Applies only to oral form of ... colestipol. colestipol decreases levels of minocycline by inhibition of GI absorption. Applies only to oral form of both agents ...
http://reference.medscape.com/drug/342549
Psyllium Information | Mount Sinai - New YorkPsyllium Information | Mount Sinai - New York
... medications called bile acid sequestrants may help further lower cholesterol levels and may reduce side effects of colestipol. ...
https://www.mountsinai.org/health-library/supplement/psyllium
All About Cholesterol-Lowering Medicines - AHealthyMe - Blue Cross Blue Shield of Massachusetts
Bile acid sequestrants. These include cholestyramine, colesevelam, and colestipol. These medicines also lower LDL, although ...
https://www.ahealthyme.com/YourFamily/Women/56,2822
Human Metabolome DatabaseHuman Metabolome Database
Colestipol. Effect. Decrease. Biospecimen. Blood. P-value. Not Available. Beta Estimator. Not Available. Percent Change. Not ... Stein EA, Heimann KW: Colestipol, clofibrate, cholestyramine and combination therapy in the treatment of familial ...
https://hmdb.ca/metabolomic_categories/2360
Nuzyra (omadacycline) dosing, indications, interactions, adverse effects, and moreNuzyra (omadacycline) dosing, indications, interactions, adverse effects, and more
colestipol. colestipol will decrease the level or effect of omadacycline by inhibition of GI absorption. Applies only to oral ... colestipol. Monitor Closely (1)colestipol will decrease the level or effect of omadacycline by inhibition of GI absorption. ...
https://reference.medscape.com/drug/nuzyra-omadacycline-1000262
Primary biliary cirrhosis: MedlinePlus Medical EncyclopediaPrimary biliary cirrhosis: MedlinePlus Medical Encyclopedia
Cholestyramine (or colestipol) may reduce the itching. Ursodeoxycholic acid may improve removal of bile from the bloodstream. ...
https://medlineplus.gov/ency/article/000282.htm
Bile acid sequestrant - wikidocBile acid sequestrant - wikidoc
Colestipol (Colestid). Side effects. Since bile acid sequestrants are designed to stay in the gut, they generally do not have ...
https://www.wikidoc.org/index.php/Bile_acid_sequestrant
ColestidCholestyramine or colestipolColesevelamHydrochlorideResinsSequestrantsAntacidsMedicationsEzetimibeColestyramineDecreaseCholesterolBile saltsHttpsEffectsTherapyColesevelamEffect of colestipol hydrochlorideFast does decadronActive ingredientAbsorptionBindsHydrochloride tabletsDrugsGranulesClofibrateBiliaryDoseMedicationDescriptorSerumResinDietaryInsolubleGemfibrozilLiverExposureLevelsMinorHighPatientsResultsAgentsSide effects
Colestid2
Cholestyramine or colestipol6
  • Cholestyramine (or colestipol) may reduce the itching. (medlineplus.gov)
  • A healthcare professional may prescribe medications for those experiencing moderate to severe pruritis, such as cholestyramine or colestipol. (medicalnewstoday.com)
  • Take cholestyramine or colestipol an hour before or four hours after taking hydrochlorothiazide if you also take either of those medications. (prescriptiongiant.com)
  • Separate dosing by ≥4hrs before or 4-6hrs after cholestyramine or colestipol resins. (empr.com)
  • If you are taking cholestyramine or colestipol, take it 4 to 6 hours before or 1 hours after gemfibrozil. (singhealth.com.sg)
  • If you also take certain drugs to lower your cholesterol (bile acid-binding resins such as cholestyramine or colestipol), take irbesartan/hydrochlorothiazide at least 4 hours before or at least 4 to 6 hours after these medications. (healthwarehouse.com)
Colesevelam1
  • These include cholestyramine, colesevelam, and colestipol. (ahealthyme.com)
Hydrochloride2
  • colestipol hydrochloride is a topic covered in the Taber's Medical Dictionary . (tabers.com)
  • Taber's Online , www.tabers.com/tabersonline/view/Tabers-Dictionary/734682/all/colestipol_hydrochloride. (tabers.com)
Resins1
Sequestrants2
  • Colestipol is in a class of medications called bile acid sequestrants. (medlineplus.gov)
  • These patients may benefit from bile acid sequestrants such as cholestyramine (2-4 g) and colestipol (5 g 2 or 3 times daily before meals). (medscape.com)
Antacids1
  • This includes aluminium-containing antacids, colestipol, phenobarbital and the combined oral contraceptive pill. (who.int)
Medications7
  • Unless otherwise instructed, take all other medications at least 1 hour before or 4 hours after you take colestipol because it can interfere with their absorption. (medlineplus.gov)
  • tell your doctor and pharmacist if you are allergic to colestipol, any other medications, or any of the ingredients in colestipol preparations. (medlineplus.gov)
  • Patients are titrated as tolerated to therapeutic doses of study medications (ezetimibe 10mg/day, niacin 1500mg/day, and colestipol 20gm/day). (druglib.com)
  • Colestipol may decrease your absorption of other medications. (blinkhealth.com)
  • Take your other medications as directed by your doctor, usually at least 1 hour before or 4 to 6 hours after colestipol. (blinkhealth.com)
  • Colestipol, a drug for lowering cholesterol, is another one that fights against blood pressure medications. (dummies.com)
  • MedlinePlus lists cholestyramine, colestipol, medications containing the hormone estrogen, protease inhibitors, retinoids, certain anti-psychotics and birth control pills as medications that can cause false high triglyceride readings. (livestrong.com)
Ezetimibe2
  • To compare LDL reduction compared to baseline in patients using maximum tolerated HMG CoA Reductase inhibitor (statin) therapy with adjunctive therapy with ezetimibe, colestipol, or niacin. (druglib.com)
  • Patients are randomized into one of three groups to receive ezetimibe, niacin, or colestipol in addition to current statin therapy. (druglib.com)
Colestyramine1
  • Reduced bioavailability w/ colestyramine or colestipol. (mims.com)
Decrease1
  • Colestipol is used along with diet changes to decrease the amount of fatty substances such as low-density lipoprotein (LDL) cholesterol ('bad cholesterol') in certain people with high cholesterol. (medlineplus.gov)
Cholesterol1
  • Colestipol is used along with a proper diet to lower cholesterol in the blood. (blinkhealth.com)
Bile salts1
  • Cholestyramine and colestipol are effective in sequestering bile salts in the enteric lumen. (medscape.com)
Https1
  • 2023. https://www.drugguide.com/ddo/view/Davis-Drug-Guide/51172/all/colestipol. (drugguide.com)
Effects2
  • Colestipol may cause side effects. (medlineplus.gov)
  • Beneficial effects of combined colestipol-niacin therapy on coronary atherosclerosis and coronary venous bypass grafts. (semanticscholar.org)
Therapy1
  • Stein EA, Heimann KW: Colestipol, clofibrate, cholestyramine and combination therapy in the treatment of familial hyperbetalipoproteinaemia. (hmdb.ca)
Colesevelam3
  • Bile-acid binding agents such as cholestyramine, colestipol , and colesevelam have been investigated for IBS-D. (nih.gov)
  • IF YOU ARE ALSO TAKING A BILE ACID SEQUESTRANT (such as cholestyramine, colestipol, or colesevelam), take this medicine at least 2 hours before or at least 4 hours after the bile acid sequestrant. (canadapharmacy.com)
  • Colesevelam binds to bile acids with higher affinity than colestyramine or colestipol, how fast does decadron injection work. (thesalarmy.com)
Effect of colestipol hydrochloride2
  • Although the significance of this finding has not been established in clinical studies, the elevation of the lipoprotein LpAI particle within the HDL fraction is consistent with an antiatherogenic effect of colestipol hydrochloride, even though little change is observed in HDL cholesterol (HDL-C). (nih.gov)
  • Modification of the carcinogenic process in colorectal cancer by endogenous and exogenous factors: effect of colestipol hydrochloride on tumors induced by dimethylhydrazine. (nih.gov)
Fast does decadron1
  • Not HGH, anabolic colestipol, how fast does decadron using steroids without a prescription can cause unpleasant side effects that go beyond drug charges and being labelled a cheater. (towermadness.com)
Active ingredient1
  • The active ingredient in colestipol hydrochloride tablets is micronized colestipol hydrochloride, which is a lipid lowering agent for oral use. (nih.gov)
Absorption4
  • colestipol will decrease the level or effect of mycophenolate by inhibition of GI absorption. (medscape.com)
  • colestipol decreases levels of digoxin by inhibition of GI absorption. (medscape.com)
  • It is also best taken at least four hours apart from calcium, iron, cholesterol-lowering drugs (cholestyramine, colestipol), and multivitamin tablets that contain iron, as these can decrease absorption. (btf-thyroid.org)
  • Cholestyramine, Colestipol, Neomycin cause decreased absorption of β carotene. (com.bd)
Binds2
  • Colestipol hydrochloride binds bile acids in the intestine forming a complex that is excreted in the feces. (nih.gov)
  • Colestipol binds to the bile acids in the intestine and prevents them being reabsorbed into the bloodstream. (dxline.info)
Hydrochloride tablets3
  • Colestipol hydrochloride tablets are light yellow in color and are tasteless and odorless. (nih.gov)
  • Colestipol hydrochloride tablets contain no calories. (nih.gov)
  • For example, Impax couldn't demonstrate that the manufacturing process for fenofibrate capsules was capable of controlling weight variations and didn't have data to support the temperature range used during the granulation process of colestipol hydrochloride tablets, according to the warning letter. (fdanews.com)
Drugs1
  • Available from: https://www.canada.ca/en/health-canada/services/drugs-health-products/drug-products/prescription-drug-list.html. (healthbloom.ca)
Granules3
  • Colestipol comes as tablets and granules to take by mouth. (medlineplus.gov)
  • A 65-year-old man with type IIa dyslipidemia who received flavored colestipol granules 2 scoops/day for 3 months developed asymptomatic hepatotoxicity. (nih.gov)
  • Colestipol granules in the colon: macroscopic and microscopic findings. (nih.gov)
Clofibrate1
  • Colestipol, clofibrate, and phytosterols in combined therapy of hyperlipidemia. (nih.gov)
Biliary1
  • Colestipol is a nonabsorbed bile acid sequestrant that is used a therapy of hyperlipidemia and for the pruritus of chronic liver disease and biliary obstruction. (nih.gov)
Dose3
  • In humans, less than 0.17% of a single 14 C-labeled colestipol hydrochloride dose is excreted in the urine when given following 60 days of dosing of 20 grams of colestipol hydrochloride per day. (nih.gov)
  • Low dose colestipol in adolescents with familial hypercholesterolaemia. (nih.gov)
  • OLYAEI, DEMATTOS BENNETT Atorvastatin Cholestyramine Colestipol Fluvastatin Gemfibrozil Lovastatin Pravastatin Simvastatin Starting dose forex powerpoint mgday 4 g bid 5 gbid 20 mg daily 600 poewrpoint 5 mg daily 10-40 mg daily 5-20 mg daily Maximum dose 80 mgday 24 gday 30 gday 80 mgday 600 bid 20 mgday 80 mgday 20 mgday Table 5. (binaryoptionsxpert.com)
Medication1
  • Colestipol, a cholesterol-lowering medication similar to cholestyramin, may also reduce beta-carotene levels. (stlukes-stl.com)
Descriptor1
  • Colestipol" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, MeSH (Medical Subject Headings) . (ouhsc.edu)
Serum5
  • Although colestipol hydrochloride produces an increase in the hepatic synthesis of cholesterol in man, serum cholesterol levels fall. (nih.gov)
  • Serum triglyceride levels may increase or remain unchanged in colestipol hydrochloride treated patients. (nih.gov)
  • The decline in serum cholesterol levels with colestipol hydrochloride treatment is usually evident by one month. (nih.gov)
  • When colestipol hydrochloride is discontinued, serum cholesterol levels usually return to baseline levels within one month. (nih.gov)
  • One week after discontinuing colestipol, serum transaminases fell dramatically, with some returning to normal limits. (nih.gov)
Resin2
  • Since colestipol hydrochloride is an anion exchange resin, the chloride anions of the resin can be replaced by other anions, usually those with a greater affinity for the resin than the chloride ion. (nih.gov)
  • Colestipol is a nonabsorbable resin. (nih.gov)
Dietary2
  • When carcinogen-treated animals received dietary colestipol (0.52%, w/w) from 4 weeks prior to the first injection of dimethylhydrazine until the time of death, there was an increase in the number of tumors per tumor-bearing animal to 2.23. (nih.gov)
  • In an attempt to understand the nature of this enhancement, animals were administered dietary colestipol at different times in relation to the administration of the carcinogen. (nih.gov)
Insoluble2
  • Colestipol is an insoluble, high molecular weight basic anion-exchange copolymer of diethylenetriamine and 1-chloro-2, 3-epoxypropane, with approximately 1 out of 5 amine nitrogens protonated (chloride form). (nih.gov)
  • Colestipol hydrochloride is hydrophilic, but it is virtually water insoluble (99.75%) and it is not hydrolyzed by digestive enzymes. (nih.gov)
Gemfibrozil1
  • if you are taking gemfibrozil (Lopid), take it 2 hours before or 2 hours after colestipol. (medlineplus.gov)
Liver2
  • Four weeks after colestipol was discontinued, all liver function tests were normal. (nih.gov)
  • Colestipol has not been associated with clinically apparent liver injury. (nih.gov)
Exposure1
  • pre-initiation colestipol exposure, 2.23. (nih.gov)
Levels1
  • colestipol decreases levels of lomitapide by drug binding in GI tract. (medscape.com)
Minor1
  • This graph shows the total number of publications written about "Colestipol" by people in this website by year, and whether "Colestipol" was a major or minor topic of these publications. (ouhsc.edu)
High3
  • The high molecular weight polymer in colestipol hydrochloride apparently is not absorbed. (nih.gov)
  • A high response rate to open label treatment with colestipol supports this, but placebo-controlled studies are warranted. (bmj.com)
  • The bile acid sequestrants (cholestyramine, colestipol) and nicotinic acid have been the mainstays of pharmacologic therapy for patients with phenotype(s) IIA and IIB hyperlipoproteinemia, but produce symptomatic side effects in a high percentage of patients. (ccjm.org)
Patients1
  • Combination therapy with colestipol and psyllium mucilloid in patients with hyperlipidemia. (nih.gov)
Results1
  • Treatment with colestipol results in a significant increase in lipoprotein LpAI. (nih.gov)
Agents1
  • Since colestipol has the capacity to bind a number of chemical agents, the different biological effects probably reflect the multifactorial nature of colorectal cancer with the end result dependent on the balance between opposing factors. (nih.gov)
Side effects1