Clorazepate Dipotassium
Nordazepam
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A stand-alone drug plan offered by insurers and other private companies to beneficiaries that receive their Medicare Part A and/or B benefits through the Original Medicare Plan. It includes Medicare Private Fee-for-Service Plans that do not offer prescription drug coverage and Medicare Cost Plans offering Medicare prescription drug coverage. The plan was enacted as the Medicare Prescription Drug, Improvement and Modernization Act of 2003 with coverage beginning January 1, 2006.
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Federal program, created by Public Law 89-97, Title XVIII-Health Insurance for the Aged, a 1965 amendment to the Social Security Act, that provides health insurance benefits to persons over the age of 65 and others eligible for Social Security benefits. It consists of two separate but coordinated programs: hospital insurance (MEDICARE PART A) and supplementary medical insurance (MEDICARE PART B). (Hospital Administration Terminology, AHA, 2d ed and A Discursive Dictionary of Health Care, US House of Representatives, 1976)
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The Balanced Budget Act (BBA) of 1997 establishes a Medicare+Choice program under part C of Title XVIII, Section 4001, of the Social Security Act. Under this program, an eligible individual may elect to receive Medicare benefits through enrollment in a Medicare+Choice plan. Beneficiaries may choose to use private pay options, establish medical savings accounts, use managed care plans, or join provider-sponsored plans.
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Acridine Orange
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Theoretical construct used in applied mathematics to analyze certain situations in which there is an interplay between parties that may have similar, opposed, or mixed interests. In a typical game, decision-making "players," who each have their own goals, try to gain advantage over the other parties by anticipating each other's decisions; the game is finally resolved as a consequence of the players' decisions.
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Descriptions of specific amino acid, carbohydrate, or nucleotide sequences which have appeared in the published literature and/or are deposited in and maintained by databanks such as GENBANK, European Molecular Biology Laboratory (EMBL), National Biomedical Research Foundation (NBRF), or other sequence repositories.
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An Act prohibiting a health plan from establishing lifetime limits or annual limits on the dollar value of benefits for any participant or beneficiary after January 1, 2014. It permits a restricted annual limit for plan years beginning prior to January 1, 2014. It provides that a health plan shall not be prevented from placing annual or lifetime per-beneficiary limits on covered benefits. The Act sets up a competitive health insurance market.
Amlodipine
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A class of drugs whose main indications are the treatment of hypertension and heart failure. They exert their hemodynamic effect mainly by inhibiting the renin-angiotensin system. They also modulate sympathetic nervous system activity and increase prostaglandin synthesis. They cause mainly vasodilation and mild natriuresis without affecting heart rate and contractility.
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Drugs used in the treatment of acute or chronic vascular HYPERTENSION regardless of pharmacological mechanism. Among the antihypertensive agents are DIURETICS; (especially DIURETICS, THIAZIDE); ADRENERGIC BETA-ANTAGONISTS; ADRENERGIC ALPHA-ANTAGONISTS; ANGIOTENSIN-CONVERTING ENZYME INHIBITORS; CALCIUM CHANNEL BLOCKERS; GANGLIONIC BLOCKERS; and VASODILATOR AGENTS.
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A methylxanthine naturally occurring in some beverages and also used as a pharmacological agent. Caffeine's most notable pharmacological effect is as a central nervous system stimulant, increasing alertness and producing agitation. It also relaxes SMOOTH MUSCLE, stimulates CARDIAC MUSCLE, stimulates DIURESIS, and appears to be useful in the treatment of some types of headache. Several cellular actions of caffeine have been observed, but it is not entirely clear how each contributes to its pharmacological profile. Among the most important are inhibition of cyclic nucleotide PHOSPHODIESTERASES, antagonism of ADENOSINE RECEPTORS, and modulation of intracellular calcium handling.
Analgesics, Non-Narcotic
S-16924 [(R)-2-[1-[2-(2,3-dihydro-benzo[1,4]dioxin-5-yloxy)-ethyl]- pyrrolidin-3yl]-1-(4-fluorophenyl)-ethanone], a novel, potential antipsychotic with marked serotonin1A agonist properties: III. Anxiolytic actions in comparison with clozapine and haloperidol. (1/16)
S-16924 is a potential antipsychotic that displays agonist and antagonist properties at serotonin (5-HT)1A and 5-HT2A/2C receptors, respectively. In a pigeon conflict procedure, the benzodiazepine clorazepate (CLZ) increased punished responses, an action mimicked by S-16924, whereas the atypical antipsychotic clozapine and the neuroleptic haloperidol were inactive. Similarly, in a Vogel conflict paradigm in rats, CLZ increased punished responses, an action shared by S-16924 but not by clozapine or haloperidol. This action of S-16924 was abolished by the 5-HT1A antagonist WAY-100,635. Ultrasonic vocalizations in rats were inhibited by CLZ, S-16924, clozapine, and haloperidol. However, although WAY-100,635 abolished the action of S-16924, it did not affect clozapine and haloperidol. In a rat elevated plus-maze, CLZ, but not S-16924, clozapine, and haloperidol, increased open-arm entries. Like CLZ, S-16924 increased social interaction in rats, whereas clozapine and haloperidol were inactive. WAY-100,635 abolished this action of S-16924. CLZ, S-16924, clozapine, and haloperidol decreased aggressive interactions in isolated mice, but this effect of S-16924 was not blocked by WAY-100, 635. All drugs inhibited motor behavior, but the separation to anxiolytic doses was more pronounced for S-16924 than for CLZ. Finally, in freely moving rats, CLZ and S-16924, but not clozapine and haloperidol, decreased dialysis levels of 5-HT in the nucleus accumbens: this action of S-16924 was blocked by WAY-100,165. In conclusion, in contrast to haloperidol and clozapine, S-16924 possessed a broad-based profile of anxiolytic activity at doses lower than those provoking motor disruption. Its principal mechanism of action was activation of 5-HT1A (auto)receptors. (+info)Use of micellar mobile phases for the chromatographic determination of clorazepate, diazepam, and diltiazem in pharmaceuticals. (2/16)
An ODS-2 column, a micellar mobile phase of high elution strength containing 0.1M sodium dodecyl sulfate and 3% (v/v) butanol, and ultraviolet detection at 230 nm are used for the determination of either of two benzodiazepines (clorazepate and diazepam) and a benzothiazepine (diltiazem) in pharmaceuticals. The procedure is shown to be competitive against conventional chromatography with methanol-water mobile phases, especially for diltiazem. The composition of the micellar mobile phase is selected using a predictive strategy based on an accurate retention model and assisted by computer simulation. Calibration graphs are linear at least in the 2.5 to 20 microg/mL, 4 to 20 microg/mL, and 5 to 40 microg/mL ranges for clorazepate, diazepam, and diltiazem, respectively. The intra- and interday repeatabilities (%) are clorazepate (1.7, 5.2), diazepam (0.43, 3.7), and diltiazem (0.36, 3.1). Limits of detection are well below the concentrations of the drugs found in the commercial pharmaceutical preparations analyzed. The drug contents evaluated with the proposed procedure are compared with the declared contents given by the manufacturers. The achieved percentages of label claim are usually between 95 and 104%. (+info)Flumazenil-sensitive dose-related physical dependence in planarians produced by two benzodiazepine and one non-benzodiazepine benzodiazepine-receptor agonists. (3/16)
Two benzodiazepine (midazolam and clorazepate) and one non-benzodiazepine (zolpidem) benzodiazepine-receptor agonists produced dose-related physical dependence, as evidenced by abstinence-induced decrease in planarian locomotor velocity (pLMV) when drug-exposed planarians were placed into drug-free water, but not when they were placed into drug-containing water (i.e., an abstinence-induced withdrawal, since the effect was only obtained in the removal of drug and not in the continued presence of drug). We have previously shown that the decrease in pLMV is associated with specific and transient withdrawal signs. In the present study, the selective benzodiazepine-receptor antagonist flumazenil significantly antagonized (P<0.05), by co-application, the ability of each agonist to produce the withdrawal. These results: (1) suggest that benzodiazepine-receptor agonists, for two different chemical categories, produce dose-related physical dependence manifested as abstinence-induced withdrawal in this simple and convenient model, and (2) in the absence of cloning or radioligand binding literature, suggest a possible specific interaction site (receptor?) for these compounds in planarians. (+info)Generalized skin drug eruption to natalizumab in a patient with multiple sclerosis. (4/16)
We report a generalized skin eruption in a young man being treated with natalizumab, a new drug used in patients with multiple sclerosis. (+info)Differential behavioral profile induced by the injection of dipotassium chlorazepate within brain areas that project to the nucleus accumbens septi. (5/16)
BACKGROUND: The effect of the agonism on gamma-aminobutyric acid (GABA) receptors was studied within medial prefrontal cortex (mPFC), amygdala (AMY) and ventral hipocampus (VH) in the plus-maze test in male rats bilaterally cannulated. These structures send glutamatergic projections to the nucleus accumbens septi (NAS), in which interaction and integration between these afferent pathways has been described. In a previous study of our group, blockade of glutamatergic transmission within NAS induced an anxiolytic like effect. METHODS: Three rat groups received either saline or dipotassium chlorazepate (1 or 2 mug/1 mul solution) 15 min before testing. Time spent in the open arms (TSOA), time per entry (TPE), extreme arrivals (EA), open and closed arms entries (OAE, CAE) and relationship between open- and closed-arms quotient (OCAQ) were recorded. RESULTS: In the AMY injected group TSOA, OAE and EA were increased by the higher doses of dipotassium chlorazepate (p < 0.01). In the mPFC, TPE was decreased by both doses (p < 0.05). Injection within ventral hippocampus (VH) decreased TSOA, OAE and OCAQ with lower doses (p < 0.05). When the three studied saline groups were compared, TSOA, OAE, EA and OCAQ were enhanced in the VH group when compared to mPFC and AMY (p < 0.001). Insertion of inner canula (p < 0.001, p < 0.01, p < 0.01) and saline injection showed an increasing significant difference (p < 0.001 in all cases) with the action of guide cannula alone within VH in TSOA, OAE and EA. CONCLUSION: We conclude that the injection of dipotassium chlorazepate has a differential effect depending of the brain area, leading to facilitatory and inhibitory effects on anxiety processing. (+info)Allosteric modulation by benzodiazepine receptor ligands of the GABAA receptor channel expressed in Xenopus oocytes. (6/16)
Chick brain mRNA was isolated and injected into Xenopus oocytes. This led to the expression in the surface membrane of functional GABA-activated channels with properties reminiscent of vertebrate GABAA channels. The GABA-induced current was analyzed quantitatively under voltage-clamp conditions. Picrotoxin inhibited this current in a concentration-dependent manner with IC50 = 0.6 microM. The allosteric modulation of GABA currents by a number of drugs acting at the benzodiazepine binding site was characterized quantitatively. In the presence of the benzodiazepine receptor ligands diazepam and clorazepate, GABA responses were enhanced, and in the presence of the convulsant beta-carboline compound methyl 6,7-dimethoxy-4-ethyl-beta-carboline-3-carboxylate (DMCM), they were depressed. Maximal stimulation of the response elicited by 10 microM GABA was 160% with diazepam and 90% with clorazepate, and maximal inhibition was 42% with DMCM, 30% with methyl beta-carboline-3-carboxylate (beta-CCM), 15% with ethyl-8-fluoro-5,6-dihydro-5-methyl-6-oxo-4H-imidazo [1,5a][1,4]benzodiazepine-3-carboxylate (Ro 15-1788), and 12% with ethyl beta-carboline-3-carboxylate (beta-CCE). Half-maximal stimulation was observed with 20 nM diazepam and 390 nM clorazepate, respectively, and half-maximal inhibition with 6 nM DMCM. beta-CCM had a similar effect to DMCM, whereas beta-CCE and Ro 15-1788 showed only small inhibition at low concentrations (less than 1 microM). All the tested carboline compounds and Ro 15-1788 showed a biphasic action and stimulated GABA current at concentrations higher than 1 microM.(ABSTRACT TRUNCATED AT 250 WORDS) (+info)Clorazepate use may prevent alcohol withdrawal convulsions. (7/16)
Clorazepate dipotassium was administered orally for the five-day prophylactic treatment of potential, incipient and overt withdrawal signs and symptoms in 226 patients on admission to an inpatient alcohol treatment unit. Conservative estimates based on these patients' histories and on literature reports predicted that between 7 and 40 (3% to 18%) of these persons would be expected to have a withdrawal convulsion. No patients experienced convulsions. This complete absence of seizures suggests that clorazepate is effective in counteracting convulsive and other manifestations of the alcohol withdrawal syndrome. (+info)Single daily dose treatment of anxiety with clobazam or dipotassium clorazepate. (8/16)
1 Forty-four clinically anxious patients entered a comparative double-blind trial of clobazam 20 mg, clobazam 30 mg and dipotassium clorazepate 15 mg, all drugs given as a single dose at night. 2 Assessment by the Hamilton Anxiety Scale, Morbid Anxiety Inventory (Salkind) and a Visual Analogue Scale showed a statistically significant improvement for all treatment groups after 2 weeks, with continued improvement after a further 2 weeks. 3 Daytime drowsiness was the commonest side-effect in all treatment groups but there was a tendency for a lower incidence in patients on clobazam. There was no evidence of a dose-related incidence of drowsiness in the clobazam 20 mg and 30 mg groups. Other side-effects were few and nonspecific. 4 Clobazam is a 1,5-benzodiazepine with an elimination half-life of 18 hours. When given in single doses of 20-30 mg at night it has an equivalent effect to dipotassium clorazepate 15 mg. (+info)
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Dipotassium Phosphate Food Grade
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Clorazepate (tranxene). Another benzodiazepine. - Semantic Scholar
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Observing Plates
Canine epileptoid cramping syndrome
Diazepam and Clorazepate Dipotassium have been used successfully to alleviate cramping in some cases, but have also failed to ...
Midazolam
... continued tapering of dose with an oral long-acting benzodiazepine such as clorazepate dipotassium. When signs of tolerance to ...
List of MeSH codes (D03)
... clorazepate dipotassium MeSH D03.438.079.080.250 - estazolam MeSH D03.438.079.080.550 - medazepam MeSH D03.438.079.080.575 - ...
List of drugs: Df-Di
... dipotassium clorazepate (INN) diprafenone (INN) dipraglurant (INN) diprenorphine (INN) Diprivan diprobutine (INN) diprofene ( ...
Clorazepate
... is used in the form of a dipotassium salt. It is unusual among benzodiazepines in that it is freely soluble in ... Clorazepate is a long-acting benzodiazepine drug. Clorazepate produces the active metabolite desmethyl-diazepam, which is a ... Clorazepate, like other benzodiazepines, is widely distributed and is highly bound to plasma proteins; clorazepate also crosses ... Special precaution is required when using clorazepate in the elderly because the elderly metabolise clorazepate more slowly, ...
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CLORAZEPATE DIPOTASSIUM- clorazepate dipotassium tablet To receive this label RSS feed. Copy the URL below and paste it into ... 2. Clorazepate dipotassium tablets can cause abuse and dependence. *Do not stop taking clorazepate dipotassium tablets all of a ... Do not take clorazepate dipotassium tablets if you:. *are allergic to clorazepate dipotassium or any of the ingredients in ... Clorazepate dipotassium tablets USP contain 3.75 mg, 7.5 mg or 15 mg of clorazepate dipotassium USP. In addition, each tablet ...
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Tranxene (clorazepate dipotassium) is available in strengths of 3.75, 7.5 and 15 mg tablets. The starting dose is usually 30mg ... Tranxene (clorazepate dipotassium) is a benzodiazepine used to treat anxiety, seizure disorders, and alcohol withdrawal ... home drugs a-z list Tranxene(Clorazepate Dipotassium) side effects drug center ... Clorazepate Dipotassium) for healthcare professionals and consumers. ...
Tranxene* T-TAB® Tablets C-IV/DEA Schedule IV
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Clorazepate dipotassium C-156 1.0 mg/mL (as clorazepate) in Methanol A certified analytical reference solution suitable for use ... Clorazepate dipotassium, marketed under the brand name Tranxene®, is a prodrug for nordiazepam, which is produced as an active ... Cerilliant now offers Certified Spiking Solutions® of the benzodiazepines Delorazepam and Clorazepate dipotassium. Delorazepam ... Clorazepate dipotassium is a long-acting benzodiazepine with anxiolytic, anticonvulsant, sedative and hypnotic properties. ...
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Clorazepate dipotassium / clorazepate dipotassium NDA 071924 global drug patent coverage, generic alternatives and suppliers
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Clorazepate is used to treat anxiety disorders, partial seizures, or alcohol withdrawal symptoms. Clorazepate may also be used ... Clorazepate affects chemicals in the brain that may be unbalanced and cause anxiety or seizures. ... Clorazepate is a benzodiazepine (ben-zoe-dye-AZE-eh-peen). ... slide 1 of 16, Clorazepate Dipotassium,. 3.75 mg, round, blue, ... What is clorazepate?. Clorazepate is a benzodiazepine (ben-zoe-dye-AZE-eh-peen). Clorazepate affects chemicals in the brain ...
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Benzodiazepine6
- Chemically, clorazepate dipotassium USP is a benzodiazepine. (nih.gov)
- Tranxene (clorazepate dipotassium) is a benzodiazepine used to treat anxiety, seizure disorders , and alcohol withdrawal symptoms . (rxlist.com)
- Clorazepate dipotassium is a long-acting benzodiazepine with anxiolytic, anticonvulsant, sedative and hypnotic properties. (cerilliant.com)
- Clorazepate is a benzodiazepine (ben-zoe-dye-AZE-eh-peen). (wellspan.org)
- Clorazepate, sold under the brand name Tranxene among others, is a benzodiazepine medication. (wikipedia.org)
- Clorazepate is an unusually long-lasting benzodiazepine and serves as a majoritive prodrug for the equally long-lasting desmethyldiazepam, which is rapidly produced as an active metabolite. (wikipedia.org)
Anxiolytic2
- Clorazepate is prescribed principally in the treatment of alcohol withdrawal and epilepsy, although it is also a useful anxiolytic because of its long half-life. (wikipedia.org)
- Itil TM, Saletu B, Marasa J (1972a) Digital computer analyzed sleep electroencephalogram (sleep prints) in predicting anxiolytic properties of clorazepate dipotassium (tranxene). (springer.com)
Benzodiazepines5
- Pharmacologically, clorazepate dipotassium has the characteristics of the benzodiazepines. (nih.gov)
- Cerilliant now offers Certified Spiking Solutions® of the benzodiazepines Delorazepam and Clorazepate dipotassium. (cerilliant.com)
- However, tolerance to the active metabolite of clorazepate may occur more slowly than with other benzodiazepines. (wikipedia.org)
- All sedatives or hypnotics e.g. other benzodiazepines, barbiturates, antiepileptic drugs, alcohol, antihistamines, opiates, neuroleptics, sleep aids are likely to magnify the effects of clorazepate on the central nervous system. (wikipedia.org)
- Clorazepate is a member of the group of drugs called benzodiazepines. (drugwiki.net)
3.75 mg2
Diazepam1
- Diazepam and Clorazepate Dipotassium have been used successfully to alleviate cramping in some cases, but have also failed to help in other cases. (wikipedia.org)
Withdrawal5
- Clorazepate dipotassium tablets are indicated for the symptomatic relief of acute alcohol withdrawal. (nih.gov)
- Withdrawal symptoms (similar in character to those noted with barbiturates and alcohol) have occurred following abrupt discontinuance of clorazepate. (nih.gov)
- Do not stop using clorazepate suddenly , or you could have unpleasant withdrawal symptoms. (wellspan.org)
- Clorazepate is used to treat anxiety disorders, partial seizures, or alcohol withdrawal symptoms. (wellspan.org)
- Clorazepate Dipotassium 15mg pills/tablets:,Clorazepate Dipotassium treats anxiety, trouble sleeping, symptoms of alcohol withdrawal, and certain types of epilepsy (seizures). (ukresearchchemicals.co.uk)
Tablets5
- Clorazepate dipotassium tablets are indicated as adjunctive therapy in the management of partial seizures. (nih.gov)
- The effectiveness of clorazepate dipotassium tablets in long-term management of anxiety, that is, more than 4 months, has not been assessed by systematic clinical studies. (nih.gov)
- Clorazepate dipotassium tablets are contraindicated in patients with a known hypersensitivity to the drug and in those with acute narrow angle glaucoma. (nih.gov)
- Tranxene (clorazepate dipotassium) is available in strengths of 3.75, 7.5 and 15 mg tablets. (rxlist.com)
- In the United States and Canada, Clorazepate is available in 3.75, 7.5, and 15 mg capsules or tablets. (wikipedia.org)
Imprint2
Nordiazepam3
- Since orally administered clorazepate dipotassium is rapidly decarboxylated to form nordiazepam, there is essentially no circulating parent drug. (nih.gov)
- Plasma levels of nordiazepam increase proportionally with Clorazepate Dipotassium dose and show moderate accumulation with repeated administration. (nih.gov)
- Clorazepate dipotassium, marketed under the brand name Tranxene®, is a prodrug for nordiazepam, which is produced as an active metabolite. (cerilliant.com)
Anticonvulsant1
- In humans, tolerance to the anticonvulsant effects of clorazepate occurs frequently with regular use. (wikipedia.org)
Discontinuation1
- Delirium has been noted from discontinuation from clorazepate. (wikipedia.org)
Controlled substance1
- Clorazepate 15 mg is classified as a Schedule 4 controlled substance under the Controlled Substance Act (CSA). (drugs.com)
Interactions1
- 2006) Interactions of buprenorphine and dipotassium clorazepate on anxiety and memory functions in the mouse. (panlab.com)
Symptoms1
- Clorazepate dipotassium is indicated for the management of anxiety disorders or for the short-term relief of the symptoms of anxiety. (nih.gov)
Urine1
- Within 10 days after oral administration of a 15 mg (50µCi) dose of 14 C-Clorazepate Dipotassium to two volunteers, 62-67% of the radioactivity was excreted in the urine and 15-19% was eliminated in the feces. (nih.gov)
Anxiety disorders1
- Clorazepate is used in the treatment of anxiety disorders and insomnia. (wikipedia.org)
Dose1
- The sustained-release formulation of clorazepate has some advantages in that, if a dose is missed, less profound fluctuations in blood plasma levels occur, which may be helpful to some people with epilepsy at risk of break-through seizures. (wikipedia.org)
Medicine1
- Some people have thoughts about suicide when taking a medicine like clorazepate. (wellspan.org)
Pregnancy1
- This is to track the outcome of the pregnancy and to evaluate any effects of clorazepate on the baby. (wellspan.org)
Effects2
- Studies in healthy men have shown that clorazepate dipotassium has depressant effects on the central nervous system. (nih.gov)
- Desmethyldiazepam is responsible for most of the therapeutic effects of clorazepate. (wikipedia.org)