An antibacterial agent that is a semisynthetic analog of LINCOMYCIN.
Substances that reduce the growth or reproduction of BACTERIA.
A bacteriostatic antibiotic macrolide produced by Streptomyces erythreus. Erythromycin A is considered its major active component. In sensitive organisms, it inhibits protein synthesis by binding to 50S ribosomal subunits. This binding process inhibits peptidyl transferase activity and interferes with translocation of amino acids during translation and assembly of proteins.
An antibiotic produced by Streptomyces lincolnensis var. lincolnensis. It has been used in the treatment of staphylococcal, streptococcal, and Bacteroides fragilis infections.
Any tests that demonstrate the relative efficacy of different chemotherapeutic agents against specific microorganisms (i.e., bacteria, fungi, viruses).
Gram-negative bacteria occurring in the lower intestinal tracts of man and other animals. It is the most common species of anaerobic bacteria isolated from human soft tissue infections.
The ability of bacteria to resist or to become tolerant to chemotherapeutic agents, antimicrobial agents, or antibiotics. This resistance may be acquired through gene mutation or foreign DNA in transmissible plasmids (R FACTORS).
A group of often glycosylated macrocyclic compounds formed by chain extension of multiple PROPIONATES cyclized into a large (typically 12, 14, or 16)-membered lactone. Macrolides belong to the POLYKETIDES class of natural products, and many members exhibit ANTIBIOTIC properties.
Infections with bacteria of the genus BACTEROIDES.
A nitroimidazole used to treat AMEBIASIS; VAGINITIS; TRICHOMONAS INFECTIONS; GIARDIASIS; ANAEROBIC BACTERIA; and TREPONEMAL INFECTIONS. It has also been proposed as a radiation sensitizer for hypoxic cells. According to the Fourth Annual Report on Carcinogens (NTP 85-002, 1985, p133), this substance may reasonably be anticipated to be a carcinogen (Merck, 11th ed).
A family of LINCOMYCIN-related glycosides that contain a pyrrolidine ring linked via an amide-bond to a pyranose moiety. Individual members of this family are defined by the arrangement of specific constituent groups on the lyncomycin molecule. Many lincosamides are ANTIBIOTICS produced by a variety STREPTOMYCES species.
The ability of microorganisms, especially bacteria, to resist or to become tolerant to chemotherapeutic agents, antimicrobial agents, or antibiotics. This resistance may be acquired through gene mutation or foreign DNA in transmissible plasmids (R FACTORS).
Infections with bacteria of the genus STREPTOCOCCUS.
A semisynthetic cephamycin antibiotic resistant to beta-lactamase.
Compounds based on ERYTHROMYCIN with the 3-cladinose replaced by a ketone. They bind the 23S part of 70S bacterial RIBOSOMES.
Potentially pathogenic bacteria found in nasal membranes, skin, hair follicles, and perineum of warm-blooded animals. They may cause a wide range of infections and intoxications.
Infections with bacteria of the genus STAPHYLOCOCCUS.
A genus of gram-negative, anaerobic, rod-shaped bacteria. Its organisms are normal inhabitants of the oral, respiratory, intestinal, and urogenital cavities of humans, animals, and insects. Some species may be pathogenic.
A naphthacene antibiotic that inhibits AMINO ACYL TRNA binding during protein synthesis.
A genus of gram-positive, facultatively anaerobic, coccoid bacteria. Its organisms occur singly, in pairs, and in tetrads and characteristically divide in more than one plane to form irregular clusters. Natural populations of Staphylococcus are found on the skin and mucous membranes of warm-blooded animals. Some species are opportunistic pathogens of humans and animals.
A species of gram-positive, coccoid bacteria isolated from skin lesions, blood, inflammatory exudates, and the upper respiratory tract of humans. It is a group A hemolytic Streptococcus that can cause SCARLET FEVER and RHEUMATIC FEVER.
A bacterium which causes mastitis in cattle and occasionally in man.
A complex of closely related aminoglycosides obtained from MICROMONOSPORA purpurea and related species. They are broad-spectrum antibiotics, but may cause ear and kidney damage. They act to inhibit PROTEIN BIOSYNTHESIS.
A chronic disorder of the pilosebaceous apparatus associated with an increase in sebum secretion. It is characterized by open comedones (blackheads), closed comedones (whiteheads), and pustular nodules. The cause is unknown, but heredity and age are predisposing factors.
Polymicrobial, nonspecific vaginitis associated with positive cultures of Gardnerella vaginalis and other anaerobic organisms and a decrease in lactobacilli. It remains unclear whether the initial pathogenic event is caused by the growth of anaerobes or a primary decrease in lactobacilli.
A specific streptogramin group B antibiotic produced by Streptomyces graminofaciens and other bacteria.
A cyclic polypeptide antibiotic complex from Streptomyces virginiae, S. loidensis, S. mitakaensis, S. pristina-spiralis, S. ostreogriseus, and others. It consists of 2 major components, VIRGINIAMYCIN FACTOR M1 and virginiamycin Factor S1. It is used to treat infections with gram-positive organisms and as a growth promoter in cattle, swine, and poultry.
Medicated dosage forms for topical application in the vagina. A cream is a semisolid emulsion containing suspended or dissolved medication; a foam is a dispersion of a gas in a medicated liquid resulting in a light, frothy mass; a jelly is a colloidal semisolid mass of a water soluble medicated material, usually translucent.
A group of antibiotics that contain 6-aminopenicillanic acid with a side chain attached to the 6-amino group. The penicillin nucleus is the chief structural requirement for biological activity. The side-chain structure determines many of the antibacterial and pharmacological characteristics. (Goodman and Gilman's The Pharmacological Basis of Therapeutics, 8th ed, p1065)
Accumulation of purulent material in tissues, organs, or circumscribed spaces, usually associated with signs of infection.
A common inhabitant of the colon flora in human infants and sometimes in adults. It produces a toxin that causes pseudomembranous enterocolitis (ENTEROCOLITIS, PSEUDOMEMBRANOUS) in patients receiving antibiotic therapy.
Infections to the skin caused by bacteria of the genus STAPHYLOCOCCUS.
Agents that soften, separate, and cause desquamation of the cornified epithelium or horny layer of skin. They are used to expose mycelia of infecting fungi or to treat corns, warts, and certain other skin diseases.
Therapy with two or more separate preparations given for a combined effect.
An acute inflammation of the INTESTINAL MUCOSA that is characterized by the presence of pseudomembranes or plaques in the SMALL INTESTINE (pseudomembranous enteritis) and the LARGE INTESTINE (pseudomembranous colitis). It is commonly associated with antibiotic therapy and CLOSTRIDIUM DIFFICILE colonization.
The ability of bacteria to resist or to become tolerant to several structurally and functionally distinct drugs simultaneously. This resistance may be acquired through gene mutation or foreign DNA in transmissible plasmids (R FACTORS).
Infections by bacteria, general or unspecified.
Substances that prevent infectious agents or organisms from spreading or kill infectious agents in order to prevent the spread of infection.
Non-susceptibility of a microbe to the action of METHICILLIN, a semi-synthetic penicillin derivative.
A semi-synthetic macrolide antibiotic structurally related to ERYTHROMYCIN. It has been used in the treatment of Mycobacterium avium intracellulare infections, toxoplasmosis, and cryptosporidiosis.
A class of natural cyclic peptide antibiotics produced by certain subspecies of STREPTOMYCES. They include two structurally unrelated components, STREPTOGRAMIN GROUP A and STREPTOGRAMIN GROUP B, which generally act synergistically to inhibit bacterial growth.
A gram-positive organism found in the upper respiratory tract, inflammatory exudates, and various body fluids of normal and/or diseased humans and, rarely, domestic animals.
A strain of Staphylococcus aureus that is non-susceptible to the action of METHICILLIN. The mechanism of resistance usually involves modification of normal or the presence of acquired PENICILLIN BINDING PROTEINS.
A macrolide antibiotic that has a wide antimicrobial spectrum and is particularly effective in respiratory and genital infections.
A penicillin derivative commonly used in the form of its sodium or potassium salts in the treatment of a variety of infections. It is effective against most gram-positive bacteria and against gram-negative cocci. It has also been used as an experimental convulsant because of its actions on GAMMA-AMINOBUTYRIC ACID mediated synaptic transmission.
Nonsusceptibility of an organism to the action of penicillins.
Infections of non-skeletal tissue, i.e., exclusive of bone, ligaments, cartilage, and fibrous tissue. The concept is usually referred to as skin and soft tissue infections and usually subcutaneous and muscle tissue are involved. The predisposing factors in anaerobic infections are trauma, ischemia, and surgery. The organisms often derive from the fecal or oral flora, particularly in wounds associated with intestinal surgery, decubitus ulcer, and human bites. (From Cecil Textbook of Medicine, 19th ed, p1688)
Derivatives of oxazolidin-2-one. They represent an important class of synthetic antibiotic agents.
A genus of gram-positive, coccoid bacteria whose organisms occur in pairs or chains. No endospores are produced. Many species exist as commensals or parasites on man or animals with some being highly pathogenic. A few species are saprophytes and occur in the natural environment.

Clindamycin plus gentamicin as expectant therapy for presumed mixed infections. (1/791)

The prevalence of obligate anaerobes was studied prospectively in 60 patients with severe sepsis of intra-abdominal, soft tissue, female genital or oropulmonary origin. In addition, the efficacy of clindamycin (for anaerobes) plus gentamicin (for aerobic bacteria, especially coliforms) as initial empiric therapy in these patients was evaluated. Among 54 patients with cultural proof of infection, anaerobic pathogens were recovered from 52%. Nineteen patients had bacteremia; Bacteroides fragilis and Klebsiella pneumoniae were the most prevalent pathogens, being isolated in five patients each. Infection was eradicated in 56 of the 60 patients (93%). Mortality related to sepsis was 7% in the entire group, 16% in patients with bacteremia and 2% in patients without bacteremia. Eighty-five percent of aerobic isolates tested were susceptible in vitro to either gentamicin or clindamycin; 97% of anaerobic isolates were inhibited by 5 mug/ml of clindamycin.  (+info)

Clindamycin suppresses endotoxin released by ceftazidime-treated Escherichia coli O55:B5 and subsequent production of tumor necrosis factor alpha and interleukin-1 beta. (2/791)

Treatment of septicemia caused by Escherichia coli with ceftazidime (CAZ) may be associated with the development of septic shock due to the release of bacterial lipopolysaccharide. We examined the suppressive effect of clindamycin (CLDM) on CAZ-induced release of endotoxin by cultured E. coli and the subsequent production of inflammatory cytokines (tumor necrosis factor alpha [TNF-alpha] and interleukin-1 beta [IL-1 beta]). E. coli ATCC 12014 was incubated in inactivated horse serum with or without CLDM for 1, 4, or 18 h, followed by the addition of CAZ and collection of the culture supernatant at 0, 1, and 2 h. The concentration of endotoxin in each sample was measured by a chromogenic Limulus test. Another portion of the culture supernatant was added to THP-1 cell culture and incubated for 4 h, and the concentrations of TNF-alpha and IL-1 beta in the supernatant were measured by an enzyme-linked immunosorbent assay. In the control group (no CLDM), CAZ administration resulted in significant increases in endotoxin, TNF-alpha, and IL-1 beta concentrations. Pretreatment of E. coli with CLDM for 4 or 18 h before the addition of CAZ significantly suppressed the concentrations of endotoxin, TNF-alpha, and IL-1 beta in a time-dependent manner. In addition, CAZ treatment transformed E. coli from rodshaped bacteria to filament-like structures, as determined by electron microscopy, while pretreatment with CLDM prevented these morphological changes. Our in vitro studies showed that CAZ-induced release of large quantities of endotoxin by E. coli could be suppressed by prior administration of CLDM.  (+info)

Antibiotic penetrance of ascitic fluid in dogs. (3/791)

Antibiotic concentrations in ascitic fluid after parenteral therapy may be important in the treatment of peritonitis. We have created ascites in dogs by partial ligation of the inferior vena cava. Ascitic fluid volume was measured at the time each antibiotic was administered. Nine antibiotics were studied in the same three dogs. Antibiotic concentration in ascitic fluid was found to vary inversely with ascites volume. Percentage of penetration (ratio of ascites peak to serum peak x100) ranged from 5.8 to 65% among the drugs studied. Only metronidazole showed a statistically significant higher percentage of penetration than other antimicrobials. Concentrations in ascitic fluid after single doses of cephalothin (15 mg/kg) and the aminoglycosides (2 mg/kg, gentamicin and tobramycin; 7.5 mg/kg, amikacin and kanamycin) did not exceed the minimum inhibitory concentration of many gram-negative rods and may justify the use of higher than usual initial parenteral doses, or possibly initial intraperitoneal administration in seriously ill patients.  (+info)

A new resistance gene, linB, conferring resistance to lincosamides by nucleotidylation in Enterococcus faecium HM1025. (4/791)

Resistance to lincomycin and clindamycin in the clinical isolate Enterococcus faecium HM1025 is due to a ribosomal methylase encoded by an ermAM-like gene and the plasmid-mediated inactivation of these antibiotics. We have cloned and determined the nucleotide sequence of the gene responsible for the inactivation of lincosamides, linB. This gene encodes a 267-amino-acid lincosamide nucleotidyltransferase. The enzyme catalyzes 3(5'-adenylation) (the adenylation of the hydroxyl group in position 3 of the molecules) of lincomycin and clindamycin. Expression of linB was observed in both Escherichia coli and Staphylococcus aureus. The deduced amino acid sequence of the enzyme did not display any significant homology with staphylococcal nucleotidyltransferases encoded by linA and linA' genes. Sequences homologous to linB were found in 14 other clinical isolates of E. faecium, indicating the spread of the resistance trait in this species.  (+info)

Antibiotic resistance of nasopharyngeal isolates of Streptococcus pneumoniae from children in Lesotho. (5/791)

Villages associated with the Lesotho Highlands Development Agency were randomized with a bias in favour of larger villages, and children < 5 years of age from cluster-randomized households in these villages were chosen for the assessment of antibiotic resistance in pneumococci. Children of the same age group attending clinics in the capital, Maseru, were selected for comparison. Nasopharyngeal cultures of Streptococcus pneumoniae from both groups of children were examined for antibiotic resistance and a questionnaire was used to assess risk factors for the acquisition of resistant strains. Carriage of penicillin- and tetracycline-resistant pneumococci was significantly higher among 196 Maseru children compared with 324 rural children (P < 0.05 and P = 0.01, respectively). Maseru children tended to visit clinics at an earlier age compared with their rural counterparts. The rural children were less exposed to antibiotics (P < 0.01), were less frequently hospitalized (P < 0.001), and rarely attended day care centres (P < 0.001). The very low incidence of antibiotic resistance in rural Lesotho and the higher incidence in Maseru are in stark contrast with the much higher frequencies found in the Republic of South Africa, many European countries, and the USA.  (+info)

In-vitro susceptibilities of species of the Bacteroides fragilis group to newer beta-lactam agents. (6/791)

The in-vitro activities of imipenem and four beta-lactam-beta-lactamase inhibitor combinations were tested against 816 strains of the Bacteroides fragilis group, and compared with other anti-anaerobic agents. None of the strains was resistant to metronidazole, and only one was resistant to chloramphenicol. Mezlocillin and piperacillin were moderately active, while clindamycin was the least active. Rates of resistance varied between various species. The new beta-lactam agents tested showed excellent activity; piperacillin-tazobactam and imipenem were the most active. The emergence of strains that are resistant to these agents, observed in this study, suggests there is a need to perform periodic antimicrobial susceptibility tests.  (+info)

Distribution of mefE and ermB genes in macrolide-resistant strains of Streptococcus pneumoniae and their variable susceptibility to various antibiotics. (7/791)

From February to October 1995, 62 erythromycin-resistant strains of Streptococcus pneumoniae isolated at Yamanashi Red Cross Hospital were tested to determine their susceptibility to various macrolides, subjected to resistance induction tests by the disc diffusion method and analysed for genes encoding resistance to macrolides (ermB and mefE). On the basis of resistance induction testing, the isolates were classified as having either inducible (59.7%) or non-inducible (40.3%) macrolide resistance. The ermB gene was always detected in resistance-inducible type isolates, either alone or in combination with mefE. The mefE gene alone was found only in non-inducible type isolates. Isolates with non-inducible resistance (those with only the mefE gene) had an intermediate level of resistance to 14-membered macrolides, and were susceptible to rokitamycin, a 16-membered macrolide. According to NCCLS guidelines, 9.6% of S. pneumoniae strains were judged to be susceptible to penicillin, 62.9% of reduced susceptibility and 27.4% penicillin resistant. No correlation was detected between the presence of particular macrolide-resistance genes (ermB, ermB + mefE, or mefE) and resistance to penicillin G.  (+info)

Further evolution of a strain of Staphylococcus aureus in vivo: evidence for significant inactivation of flucloxacillin by penicillinase. (8/791)

A strain of Staphylococcus aureus (no. FAR4) has been isolated at intervals, for 32 months, from the sputum of a patient with cystic fibrosis of the lung. Changes in the properties of isolates of this strain over the first 18 months have been reported previously (Lacey et al., 1973 and 1974). During the last 14 months (May 1973 to July 1974), further evolution has occurred to produce a total of 31 distinct phenotypes. Recent changes are as follows. 1. The ability of isolates to produce penicillinase in vitro was closely correlated with flucloxacillin therapy. Inactivation of flucloxacillin by penicillinase was demonstrated by diffusion testing (but not MIC determination) in vitro and may have occurred to a significant extent in vivo. 2. Lincomycin-resistant mutants slowly disappeared from the sputum after the termination of clindamycin therapy. 3. All of the recent isolates were resistant to erythromycin, possibly because of the linkage of the genes coding for erythromycin resistance with those coding for the production of delta-haemolysin; delta-haemolysin may be an important "virulence factor".  (+info)

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The phosphate salt form of clindamycin, a semi-synthetic, chlorinated broad spectrum antibiotic produced by chemical modification of lincomycin. Clindamycin phosphate is used in topical preparations.
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Dalacin-C (Clindamycin Phosphate) is a type of antibiotic, which is used to treat various bacterial infections, such as those affecting the lungs, blood, skin, internal organs and elsewhere in the body. This medication is in a class of drugs known as lincomycin antibiotics, which treat infections by fighting the bacteria that cause infections.
If you have acne, a doctor may recommend clindamycin phosphate lotion. This eMedTV resource further discusses how this medication can help treat certain bacterial skin infections and describes some side effects that may occur during treatment.
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INDICATIONS Clindamycin is indicated in the treatment of serious infections caused by susceptible anaerobic bacteria. Clindamycin is also indicated in the treatment of serious infections due to susceptible strains of streptococci, pneumococci, and staphylococci. Its use should be reserved for penicillin-allergic patients or other patients for whom, in the judgment of the physician, a penicillin is inappropriate. Because of the risk of colitis, as described in the WARNING box, before selecting clindamycin the physician should consider the nature of the infection and the suitability of less toxic alternatives (eg, erythromycin).. INSTRUCTIONS Take clindamycin exactly as prescribed by your doctor.. ...
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TY - JOUR. T1 - The use of parenteral clindamycin in the treatment of obstetric gynecologic patients with severe infections. A comparison of a clindamycin kanamycin combination with penicillin kanamycin. AU - Ledger, W. J.. AU - Kriewall, T. J.. AU - Sweet, Richard L. AU - Fekety, F. R.. PY - 1974. Y1 - 1974. N2 - Using a system of random selection in patients with severe obstetric gynecologic infections, parenteral penicillin kanamycin was given to 23 women and clindamycin kanamycin was given to 21. Therapeutic response to the two antibiotic regimens was similar; no significant differences were noted in either the posttreatment fever index or the numbers of women requiring other therapy. There was a failure of response to the penicillin kanamycin regimen in those women with Bacteroides fragilis infection and to the clindamycin kanamycin regimen in some of the women with an Enterococcus infection. The implications of these findings for the future selection of antibiotics for patients with severe ...
INDICATIONS Clindamycin is indicated in the treatment of serious infections caused by susceptible anaerobic bacteria. Clindamycin is also indicated in the treatment of serious infections due to susceptible strains of streptococci, pneumococci, and staphylococci. Its use should be reserved for penicillin-allergic patients or other patients for whom, in the judgment of the physician, a penicillin is inappropriate. Because of the risk of colitis, as described in the WARNING box, before selecting clindamycin the physician should consider the nature of the infection and the suitability of less toxic alternatives (eg, erythromycin).. INSTRUCTIONS Take clindamycin exactly as prescribed by your doctor.. ...
If the symptoms of your infection do not improve within a few days, or if they become worse, tell your doctor.. If you get severe diarrhoea, tell your doctor, pharmacist or nurse immediately. Do this even if it occurs several weeks after the clindamycin injection has been stopped. Diarrhoea may mean that you have a serious condition affecting your bowel. You may need urgent medical care.. Do not take any medicines for diarrhoea without first checking with your doctor.. If you get a severe skin rash tell your doctor immediately. Do this even if the rash occurs after CLINDAMYCIN MYLAN Injection has been stopped. A severe skin rash may mean you are having an allergic reaction to CLINDAMYCIN MYLAN Injection. You may need urgent medical care.. If you get a sore, white mouth or tongue while being given this medicine or soon after clindamycin injection has been stopped, tell your doctor. Also tell your doctor if you get vaginal itching or discharge. This may mean you have a fungal/yeast infection ...
To the Editor: We report the appearance of erythromycin and inducible clindamycin resistance in the south-west Pacific strain of non-multiresistant methicillin-resistant Staphylococcus aureus, which has recently appeared in eastern Australia. Infections occur predominantly in Polynesian people and are usually community-acquired. Most strains belong to Western Samoan phage patterns (WSPP1 or WSPP2) and pulsotype A when typed by pulsed-field gel electrophoresis.1,2 These strains are resistant to all β-lactams, but are usually susceptible to erythromycin, clindamycin, gentamicin, tetracycline, trimethoprim-sulfamethoxazole and ciprofloxacin. Although most of these antibiotics would not be recommended for therapy,3 clindamycin has been recommended for non-parenteral treatment of soft-tissue and bone infections, as it is efficacious in treating similar infections caused by methicillin-susceptible S. aureus.4. ...
Can I Take Clindamycin While Breastfeeding. Clindamycin use while Breastfeeding . Includes possible effects on breastfed infants and lactation. Clindamycin - InfantRisk Forums (cleocin) 300 mg 3 times a day. I was told by the ER doctor that I should NOT breastfeed while on this medication and to pump and dump. My baby is 9 weeks old and has never had a bottle, he hates it and its torture not being able to nurse. Clindamycin And Breastfeeding - Lactation Consultant NYC Clindomycin does pass through breast milk. Although the manufacturers generally recommend that this drug should be avoided while breastfeeding. Continued breastfeeding should be encouraged while you have Mastitis and are taking antibiotics, unless your doctor has told you otherwise. Consulting nbsp; Clindamycin and Pregnancy MotherToBaby Most studies on clindamycin have looked at oral or vaginal use during the second or third trimester. I am breastfeeding. Can I take clindamycin? Yes. You should not stop taking antibiotics without ...
There are many different factors that cause acne. So combination treatment using different medications that can address these different factors is commonly used to treat acne. Fixed-dose combination clindamycin phosphate 1.2% and tretinoin 0.025% gel and benzoyl peroxide 6% foaming cloths are both used to treat acne. This study will evaluate the use of the medications when used together ...
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How helpful was it? Clindamycin is also available as an oral solution, topical foam, topical gel, topical lotion, topical swab, topical solution, vaginal suppository, and vaginal cream. Check symptom Check drugs Compare drugs. Since the time I have been taking them I have been having trouble sleeping and having really bad nightmares.. Here is a quick synopsis in the past month I have taken clindamycin no side effects Cipro for sinus infection horrible digestive effects and erythromycin stop taking that as well due to side effects, mainly diarhhea, fatigue and gas.. It is a very strong antibiotic, but it does not make a person sleepy. Had two cough drops and finally fell asleep for a couple of hours while sitting up. I experienced very severe pain in both joints of my jaw about one hour after taking clindymicin on the third day of treatment. Thereafter, serious muscle spasms in both my thighs.. Everytime they give me that it all goes away and I feel great for several months. Could the clindamycin ...
CLINDAMYCIN PALMITATE HYDROCHLORIDE, INDICATIONS AND USAGE Clindamycin palmitate hydrochloride for oral solution is indicated in the treatment of serious infections caused by susceptible anaerobic bacteria. Clindamycin is also indicated
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Is Urticaria a common side effect of Clindamycin? View Urticaria Clindamycin side effect risks. Female, 30 years of age, weighting 192.0 lb, was diagnosed with infection and took Clindamycin 1 Pill, Three Times Daily, Taken By Mouth.
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The safety and scientific validity of this study is the responsibility of the study sponsor and investigators. Listing a study does not mean it has been evaluated by the U.S. Federal Government. Read our disclaimer for details ...
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The skin is the bodys largest organ, and it contains literally millions of the super small holes which are its pores. Each pore contains a special oil gland that secretes a substance called sebum, the oil needed to keep the skin properly lubricated. The problem is that sometimes these glands make too much sebum, and when this excess gets mixed together with dead skin cells and other dirt and grime this plugs the pore. Depending on a persons age (and other factors), when pores become clogged up like this the result is acne, blackheads, pores of winer, cysts, and other skin problems.. Cleocin Gel (clindamycin phosphate) is a cheap and effective prescription acne medication thats suitable for people of all ages who have sebaceous gland related skin problems. It can be used to treat acne breakouts and newly developing cysts when they occur. As for those who suffer from chronic skin problems, applying Cleocin Gel twice a day controls the growth of bacteria responsible for acne and other skin ...
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Forty-four (71.0%) patients experienced SSTI recurrence at a median of 135.5 days (range: 24-1116 days), with a median follow-up time of 190.5 days (range: 24-1495 days). Fifty percent of all patients had experienced SSTI recurrence by 281.5 days. Twelve (27.3%) patients had SSTI recurrence at the same site as the initial infection; no correlation was observed between anatomic site of initial infection and same-site SSTI recurrence. A culture yielded MRSA at the time of follow-up in 14 (31.8%) SSTI recurrences; no other pathogens were isolated. All these 14 recurrent isolates were susceptible to trimethoprim-sulfamethoxazole and rifampin. Five (35.7%) recurrent isolates were constitutively resistant to clindamycin, whereas 1 recurrent isolate displayed intermediate susceptibility to clindamycin. Two of the recurrent isolates were cultured from patients who had initial isolates resistant to clindamycin. Of the 3 isolates with acquired clindamycin resistance, only 1 was from a patient who received ...
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Hi, I am writing this review because the reports, when I got sick with the tablets myself, have helped me a lot, and maybe I can help with my experience: After my whitening tooth operation (a wound had become inflamed) I have Clindamycin 600 Get prescribed 3 times a day. On the 5th day I first got cramps and slimy diarrhea, a day later I had blood in my stool for the first time. I also had a strange chemical taste in my mouth from the pills and got tired and tired. I stopped the antibiotic after 6 days. The bleeding got worse. I went to the specialist, because I had already read on the Internet that many others get through Clinda intestinal problems. Still without findings I got by suspicion on Clostridium difficile metronidazole 400 (3xtäglich). The stool sample confirmed the suspicion. However, the symptoms worsened further with the use of metronidazole. After a week of metronidazole, I could barely stand up. I had to go to the bathroom more than 20 times a day and it was always a lot of ...
Feddock CA. In small skin abscesses, clindamycin or trimethoprim-sulfamethoxazole after incision and drainage increased cures. Ann Intern Med. 2017;167:JC39. doi: 10.7326/ACPJC-2017-167-8-039. Download citation file:. ...
Streptococci and S. aureus are the most common pathogens identified in patients with cellulitis (table 2), and accumulating evidence from prospective convalescent serology studies suggests that , 70% are caused by streptococci.4,79 Atypical pathogens can be observed in patients with selected conditions (table 3). In contrast to diabetic foot infections, diabetic non-foot infections are generally not caused by atypical pathogens.87 In the Netherlands, the preferred small spectrum agent covering both methicillin-susceptible S. aureus and beta-haemolytic streptococci is flucloxacillin. Confirmed streptococcal infections can be treated with benzylpenicillin or feneticillin. Co-amoxiclav and clindamycin are alternative options. Clindamycin is recommended in case of beta-lactam allergies, and inhibits streptococcal and staphylococcal toxin production. Clindamycin is also thought to have better tissue penetration than beta-lactams. However, clindamycin is highly concentrated intracellularly, and ...
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Safety Data Sheet Fiche de Données de Sécurité Clindamycin is an antibiotic used for the treatment of a number of bacterial infections, including bone or joint infections, pelvic inflammatory disease, strep throat, pneumonia, middle ear infections, and endocarditis. It can also be used to treat acne, and some cases of
Clindamycin, given in the form of oral antirobe caps, are used to treat periodontal disease, soft-tissue infections, and osteomyelitis in dogs.
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"Clindamycin" (PDF). Davis. 2017. Retrieved March 24, 2017.[permanent dead link] Bernhard AE, Field KG (October 2000). "A PCR ... It is susceptible to clindamycin. An alternative fecal indicator organism, Bacteroides, has been suggested because they make up ... Löfmark S, Jernberg C, Jansson JK, Edlund C (December 2006). "Clindamycin-induced enrichment and long-term persistence of ...
For most infections, clindamycin and penicillin treatment is recommended. Further, a cocktail of antimicrobials targeting ...
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If a 'D' shape is formed around the clindamycin disk then the isolate is reported as resistant to clindamycin. This occurs due ... To test for clindamycin resistance certain strains of Staphylococcus aureus bacteria with natural resistance to erythromycin ... The two antimicrobial disks contain erythromycin and clindamycin and are placed about 20 mm apart. This is called a D-zone test ... Megged O, Assous M, Weinberg G, Schlesinger Y (2013). "Inducible clindamycin resistance in β-hemolytic streptococci and ...
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This is why adding an antibiotic like clindamycin, which acts ribosomally, kills some of the bacteria and returns them to the ... Stevens, DL; Gibbons, AE; Bergstrom, R; Winn, V (July 1988). "The Eagle effect revisited: efficacy of clindamycin, erythromycin ...
Application of adapalene gel to the skin 3-5 minutes before application of clindamycin enhances penetration of clindamycin into ... Adapalene has been shown to enhance the efficacy of topical clindamycin, although adverse effects are also increased. ... Jain GK, Ahmed FJ (2007). "Adapalene pretreatment increases follicular penetration of clindamycin: in vitro and in vivo studies ... Jain, GauravK; Ahmed, FarhanJ (2007). "Adapalene pretreatment increases follicular penetration of clindamycin: In vitro and in ...
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Cook-Bolden FE, Barba A, Halder R, Taylor S (2004). "Twice-daily applications of benzoyl peroxide 5%/clindamycin 1% gel versus ...
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The topically used clindamycin phosphate is a phosphate-ester prodrug of clindamycin. Clindamycin has a primarily ... Topical application of clindamycin phosphate can be used to treat mild to moderate acne. The use of clindamycin in conjunction ... The combination of clindamycin and quinine is the standard treatment for severe babesiosis. Clindamycin may also be used to ... If the area of inhibition around the clindamycin disk is "D" shaped, the test result is positive and clindamycin should not be ...
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  • Most aerobic Gram-negative bacteria (such as Pseudomonas, Legionella, Haemophilus influenzae and Moraxella) are resistant to clindamycin, as are the facultative anaerobic Enterobacteriaceae. (wikipedia.org)
  • 64 μg/ml When testing a Gram-positive culture for sensitivity to clindamycin, it is common to perform a "D-Test" to determine if there is a macrolide-resistant subpopulation of bacteria present. (wikipedia.org)
  • This test is necessary because some bacteria express a phenotype known as MLSB, in which susceptibility tests will indicate the bacteria are susceptible to clindamycin, but in vitro the pathogen displays inducible resistance. (wikipedia.org)
  • To perform a D-test, an agar plate is inoculated with the bacteria in question and two drug-impregnated disks (one with erythromycin, one with clindamycin) are placed 15-20 mm apart on the plate. (wikipedia.org)
  • Clindamycin is used for treatment of serious infections caused by susceptible bacteria. (medicinenet.com)
  • Clindamycin causes Clostridium difficile associated diarrhea (CDAD) because it can alter the normal bacteria in the colon and encourage overgrowth Clostridium difficile , a bacteria which causes inflammation of the colon (pseudomembranous colitis ). (medicinenet.com)
  • Clindamycin vaginal (for use in the vagina) is used to treat vaginal infections caused by bacteria. (wellspan.org)
  • Clindamycin HCl treats infections caused by bacteria, not viruses, and will not work for viral infections like the common cold or flu. (helprx.info)
  • Clindamycin is an antibiotic useful for the treatment of a number of bacterial infections. (wikipedia.org)
  • Clindamycin is an antibiotic used for treating serious infections. (medicinenet.com)
  • Clindamycin (Cleocin) is an antibiotic prescribed to fight serious bacterial infections, for example, pneumonitis, lung abscess, ovarian abscess, endometritis, and serious skin and soft tissue infections. (medicinenet.com)
  • Clindamycin was prescribed for respiratory tract infections in 38.9% of patients and for prophylaxis in surgery in 25.4% (71% were contaminated during surgery). (medscape.com)
  • Clindamycin (Cleocin) is a lincosamide used to treat serious skin and soft tissue staphylococcal infections. (medscape.com)
  • Clindamycin will not work against viral infections such as colds or flu. (nps.org.au)
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  • The use of parenteral clindamycin in the treatment of obstetric gynecologic patients with severe infections. (elsevier.com)
  • Using a system of random selection in patients with severe obstetric gynecologic infections, parenteral penicillin kanamycin was given to 23 women and clindamycin kanamycin was given to 21. (elsevier.com)
  • Clindamycin phosphate, a lincosamide, is a water soluble ester of the semi-synthetic antibiotic produced by a 7(S)-chloro-substitution of the 7(R)-hydroxyl group of the parent antibiotic lincomycin. (rxlist.com)
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  • This leaflet answers some common questions about clindamycin injection. (nps.org.au)
  • Your doctor has weighed the risks of you taking clindamycin injection against the benefits this medicine is expected to have for you. (nps.org.au)
  • CLINDAMYCIN MYLAN injection is recommended for patients who are allergic to penicillin or patients for whom, in the judgment of the doctor, penicillin is inappropriate. (nps.org.au)
  • Your doctor may have prescribed CLINDAMYCIN MYLAN injection for another reason. (nps.org.au)
  • Clindamycin should not be used during the first trimester of pregnancy unless it is clearly needed because it has not been properly evaluated during the first trimester of pregnancy. (medicinenet.com)
  • Clindamycin is a pregnancy category C medication. (medscape.com)
  • You should not take clindamycin HCL if you have certain medical conditions, including the intestinal condition Clostridium difficile-associated diarrhea. (helprx.info)
  • Clindesse (clindamycin phosphate) does not comply with the pH test of the USP monograph for clindamycin phosphate vaginal cream. (rxlist.com)
  • Clindesse is a semi-solid, white cream, which contains clindamycin phosphate, USP, at a concentration equivalent to 20 mg clindamycin base per gram. (rxlist.com)
  • What are the precautions when taking clindamycin phosphate (Clindesse)? (rxlist.com)
  • Clindesse (clindamycin phosphate) is indicated for the treatment of bacterial vaginosis (formerly referred to as Haemophilus vaginitis, Gardnerella vaginitis, nonspecific vaginitis, Corynebacterium vaginitis, or anaerobic vaginosis) in non-pregnant women. (rxlist.com)
  • The data described below reflect exposure to Clindesse (clindamycin phosphate) in 368 patients. (rxlist.com)
  • Clindesse (clindamycin phosphate) was studied in three clinical studies: placebo-controlled (n=85), active-controlled (n=263), and single-arm (n=20). (rxlist.com)
  • Adverse reactions occurred in 126 of 368 patients (34%) treated with Clindesse (clindamycin phosphate) and in 32 of 85 patients (38%) treated with placebo. (rxlist.com)
  • Adverse reactions occurring in ≥ 1% of patients receiving Clindesse (clindamycin phosphate) in the three clinical studies are shown in Table 1. (rxlist.com)
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  • Been on clindamycin since Saturday evening and this is Thursday night for an abscessed tooth. (medschat.com)
  • Patients who develop signs of pseudomembranous colitis after starting clindamycin ( diarrhea , fever , abdominal pain , and possibly shock ) should contact their physician immediately. (medicinenet.com)
  • You should not use clindamycin vaginal if you have an intestinal disorder such as colitis or Crohn's disease. (wellspan.org)
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