Citalopram: A furancarbonitrile that is one of the SEROTONIN UPTAKE INHIBITORS used as an antidepressant. The drug is also effective in reducing ethanol uptake in alcoholics and is used in depressed patients who also suffer from tardive dyskinesia in preference to tricyclic antidepressants, which aggravate this condition.Serotonin Uptake Inhibitors: Compounds that specifically inhibit the reuptake of serotonin in the brain.Antidepressive Agents, Second-Generation: A structurally and mechanistically diverse group of drugs that are not tricyclics or monoamine oxidase inhibitors. The most clinically important appear to act selectively on serotonergic systems, especially by inhibiting serotonin reuptake.Antidepressive Agents: Mood-stimulating drugs used primarily in the treatment of affective disorders and related conditions. Several MONOAMINE OXIDASE INHIBITORS are useful as antidepressants apparently as a long-term consequence of their modulation of catecholamine levels. The tricyclic compounds useful as antidepressive agents (ANTIDEPRESSIVE AGENTS, TRICYCLIC) also appear to act through brain catecholamine systems. A third group (ANTIDEPRESSIVE AGENTS, SECOND-GENERATION) is a diverse group of drugs including some that act specifically on serotonergic systems.Serotonin Plasma Membrane Transport Proteins: Sodium chloride-dependent neurotransmitter symporters located primarily on the PLASMA MEMBRANE of serotonergic neurons. They are different than SEROTONIN RECEPTORS, which signal cellular responses to SEROTONIN. They remove SEROTONIN from the EXTRACELLULAR SPACE by high affinity reuptake into PRESYNAPTIC TERMINALS. Regulates signal amplitude and duration at serotonergic synapses and is the site of action of the SEROTONIN UPTAKE INHIBITORS.Dothiepin: A tricyclic antidepressant with some tranquilizing action.Serotonin: A biochemical messenger and regulator, synthesized from the essential amino acid L-TRYPTOPHAN. In humans it is found primarily in the central nervous system, gastrointestinal tract, and blood platelets. Serotonin mediates several important physiological functions including neurotransmission, gastrointestinal motility, hemostasis, and cardiovascular integrity. Multiple receptor families (RECEPTORS, SEROTONIN) explain the broad physiological actions and distribution of this biochemical mediator.Fluoxetine: The first highly specific serotonin uptake inhibitor. It is used as an antidepressant and often has a more acceptable side-effects profile than traditional antidepressants.Paroxetine: A serotonin uptake inhibitor that is effective in the treatment of depression.Cyclohexanols: Monohydroxy derivatives of cyclohexanes that contain the general formula R-C6H11O. They have a camphorlike odor and are used in making soaps, insecticides, germicides, dry cleaning, and plasticizers.Antidepressive Agents, Tricyclic: Substances that contain a fused three-ring moiety and are used in the treatment of depression. These drugs block the uptake of norepinephrine and serotonin into axon terminals and may block some subtypes of serotonin, adrenergic, and histamine receptors. However the mechanism of their antidepressant effects is not clear because the therapeutic effects usually take weeks to develop and may reflect compensatory changes in the central nervous system.Clomipramine: A tricyclic antidepressant similar to IMIPRAMINE that selectively inhibits the uptake of serotonin in the brain. It is readily absorbed from the gastrointestinal tract and demethylated in the liver to form its primary active metabolite, desmethylclomipramine.Norepinephrine Plasma Membrane Transport Proteins: Sodium chloride-dependent neurotransmitter symporters located primarily on the PLASMA MEMBRANE of noradrenergic neurons. They remove NOREPINEPHRINE from the EXTRACELLULAR SPACE by high affinity reuptake into PRESYNAPTIC TERMINALS. It regulates signal amplitude and duration at noradrenergic synapses and is the target of ADRENERGIC UPTAKE INHIBITORS.Serotonin Antagonists: Drugs that bind to but do not activate serotonin receptors, thereby blocking the actions of serotonin or SEROTONIN RECEPTOR AGONISTS.Receptor, Serotonin, 5-HT1A: A serotonin receptor subtype found distributed through the CENTRAL NERVOUS SYSTEM where they are involved in neuroendocrine regulation of ACTH secretion. The fact that this serotonin receptor subtype is particularly sensitive to SEROTONIN RECEPTOR AGONISTS such as BUSPIRONE suggests its role in the modulation of ANXIETY and DEPRESSION.Imipramine: The prototypical tricyclic antidepressant. It has been used in major depression, dysthymia, bipolar depression, attention-deficit disorders, agoraphobia, and panic disorders. It has less sedative effect than some other members of this therapeutic group.Depressive Disorder, Major: Marked depression appearing in the involution period and characterized by hallucinations, delusions, paranoia, and agitation.Forensic Pathology: The application of pathology to questions of law.Hindlimb Suspension: Technique for limiting use, activity, or movement by immobilizing or restraining animal by suspending from hindlimbs or tails. This immobilization is used to simulate some effects of reduced gravity and study weightlessness physiology.Tryptophan Hydroxylase: An enzyme that catalyzes the hydroxylation of TRYPTOPHAN to 5-HYDROXYTRYPTOPHAN in the presence of NADPH and molecular oxygen. It is important in the biosynthesis of SEROTONIN.8-Hydroxy-2-(di-n-propylamino)tetralin: A serotonin 1A-receptor agonist that is used experimentally to test the effects of serotonin.Mianserin: A tetracyclic compound with antidepressant effects. It may cause drowsiness and hematological problems. Its mechanism of therapeutic action is not well understood, although it apparently blocks alpha-adrenergic, histamine H1, and some types of serotonin receptors.Sertraline: A selective serotonin uptake inhibitor that is used in the treatment of depression.Serotonin Receptor Agonists: Endogenous compounds and drugs that bind to and activate SEROTONIN RECEPTORS. Many serotonin receptor agonists are used as ANTIDEPRESSANTS; ANXIOLYTICS; and in the treatment of MIGRAINE DISORDERS.p-Chloroamphetamine: Chlorinated analog of AMPHETAMINE. Potent neurotoxin that causes release and eventually depletion of serotonin in the CNS. It is used as a research tool.Fluvoxamine: A selective serotonin reuptake inhibitor that is used in the treatment of DEPRESSION and a variety of ANXIETY DISORDERS.Depressive Disorder: An affective disorder manifested by either a dysphoric mood or loss of interest or pleasure in usual activities. The mood disturbance is prominent and relatively persistent.Drug Overdose: Accidental or deliberate use of a medication or street drug in excess of normal dosage.Raphe Nuclei: Collections of small neurons centrally scattered among many fibers from the level of the TROCHLEAR NUCLEUS in the midbrain to the hypoglossal area in the MEDULLA OBLONGATA.Adrenergic Uptake Inhibitors: Drugs that block the transport of adrenergic transmitters into axon terminals or into storage vesicles within terminals. The tricyclic antidepressants (ANTIDEPRESSIVE AGENTS, TRICYCLIC) and amphetamines are among the therapeutically important drugs that may act via inhibition of adrenergic transport. Many of these drugs also block transport of serotonin.Serotonin 5-HT1 Receptor Antagonists: Drugs that bind to but do not activate SEROTONIN 5-HT1 RECEPTORS, thereby blocking the actions of SEROTONIN 5-HT1 RECEPTOR AGONISTS. Included under this heading are antagonists for one or more of the specific 5-HT1 receptor subtypes.Gastric Lavage: Medical procedure involving the emptying of contents in the stomach through the use of a tube inserted through the nose or mouth. It is performed to remove poisons or relieve pressure due to intestinal blockages or during surgery.Forensic Toxicology: The application of TOXICOLOGY knowledge to questions of law.Narcotics: Agents that induce NARCOSIS. Narcotics include agents that cause somnolence or induced sleep (STUPOR); natural or synthetic derivatives of OPIUM or MORPHINE or any substance that has such effects. They are potent inducers of ANALGESIA and OPIOID-RELATED DISORDERS.Nalorphine: A narcotic antagonist with some agonist properties. It is an antagonist at mu opioid receptors and an agonist at kappa opioid receptors. Given alone it produces a broad spectrum of unpleasant effects and it is considered to be clinically obsolete.Drug and Narcotic Control: Control of drug and narcotic use by international agreement, or by institutional systems for handling prescribed drugs. This includes regulations concerned with the manufacturing, dispensing, approval (DRUG APPROVAL), and marketing of drugs.Levorphanol: A narcotic analgesic that may be habit-forming. It is nearly as effective orally as by injection.Drugs, Generic: Drugs whose drug name is not protected by a trademark. They may be manufactured by several companies.Heart Rate: The number of times the HEART VENTRICLES contract per unit of time, usually per minute.Deductibles and Coinsurance: Cost-sharing mechanisms that provide for payment by the insured of some portion of covered expenses. Deductibles are the amounts paid by the insured under a health insurance contract before benefits become payable; coinsurance is the provision under which the insured pays part of the medical bill, usually according to a fixed percentage, when benefits become payable.Sotalol: An adrenergic beta-antagonist that is used in the treatment of life-threatening arrhythmias.Heart: The hollow, muscular organ that maintains the circulation of the blood.Amiodarone: An antianginal and class III antiarrhythmic drug. It increases the duration of ventricular and atrial muscle action by inhibiting POTASSIUM CHANNELS and VOLTAGE-GATED SODIUM CHANNELS. There is a resulting decrease in heart rate and in vascular resistance.Circadian Rhythm: The regular recurrence, in cycles of about 24 hours, of biological processes or activities, such as sensitivity to drugs and stimuli, hormone secretion, sleeping, and feeding.Drug Information Services: Services providing pharmaceutic and therapeutic drug information and consultation.Drug-Related Side Effects and Adverse Reactions: Disorders that result from the intended use of PHARMACEUTICAL PREPARATIONS. Included in this heading are a broad variety of chemically-induced adverse conditions due to toxicity, DRUG INTERACTIONS, and metabolic effects of pharmaceuticals.Medication Systems: Overall systems, traditional or automated, to provide medication to patients.Patient Medication Knowledge: Patient health knowledge related to medications including what is being used and why as well as instructions and precautions.Pharmaceutical Preparations: Drugs intended for human or veterinary use, presented in their finished dosage form. Included here are materials used in the preparation and/or formulation of the finished dosage form.Verbal Behavior: Includes both producing and responding to words, either written or spoken.Cognition: Intellectual or mental process whereby an organism obtains knowledge.Neuropsychological Tests: Tests designed to assess neurological function associated with certain behaviors. They are used in diagnosing brain dysfunction or damage and central nervous system disorders or injury.Cognition Disorders: Disturbances in mental processes related to learning, thinking, reasoning, and judgment.Treatment Outcome: Evaluation undertaken to assess the results or consequences of management and procedures used in combating disease in order to determine the efficacy, effectiveness, safety, and practicability of these interventions in individual cases or series.Intelligence: The ability to learn and to deal with new situations and to deal effectively with tasks involving abstractions.

Modulation of noradrenergic neuronal firing by selective serotonin reuptake blockers. (1/558)

Using in vivo extracellular unitary recording, the effect of short term (2-day) and long-term (21-day) administration of the selective 5-HT reuptake inhibitor (SSRI) paroxetine (10 mg kg(-1) day(-1), s.c. using osmotic minipumps) was examined on the spontaneous firing activity of locus coeruleus noradrenergic neurons. Long-term but not short-term treatment significantly decreased firing activity. Thus, it appears that enhancing 5-HT neurotransmission by sustained SSRI administration leads to a reduction of the firing rate of noradrenergic neurons. The SSRI paroxetine therefore alters the activity of noradrenergic neurons with a delay that is consistent with its therapeutic action in depression and panic disorder.  (+info)

Effect of activated charcoal alone or given after gastric lavage in reducing the absorption of diazepam, ibuprofen and citalopram. (2/558)

AIMS: The efficacy of activated charcoal alone, and gastric lavage followed by charcoal in reducing the absorption of diazepam, ibuprofen and citalopram was studied in healthy volunteers. METHODS: In a randomized cross-over study with three phases, nine healthy volunteers were administered single oral doses of 5 mg diazepam, 400 mg ibuprofen and 20 mg citalopram, taken simultaneously after an overnight fast. Thirty minutes later, the subjects were assigned to one of the following treatments: 200 ml water (control), 25 g activated charcoal as a suspension in 200 ml water or gastric lavage followed by 25 g charcoal in suspension given through the lavage tube. Plasma concentrations of diazepam, ibuprofen and citalopram were determined up to 10 h. RESULTS: The AUC(0,10 h) of diazepam was reduced by 27% (P<0.05) by both charcoal alone and charcoal combined with lavage. The increase in plasma diazepam concentration from 0.5 h onwards was prevented by both interventions (P+info)

Serotonin reuptake inhibition by citalopram in rat strains differing for their emotionality. (3/558)

Acute administration of the selective serotonin (5-HT) reuptake inhibitor (SSRI), citalopram (1-10 mg/kg, i.p. 1 h before an elevated plus-maze test), to Spontaneously Hypertensive rats (SHRs), Lewis (LEW) rats, and Wistar-Kyoto (WKY) rats, i.e., rat strains differing for their emotionality, promoted anxiety, and/or hypoactivity, except in WKY rats. In the three strains, such a pretreatment increased central 5-HT levels and/or decreased 5-hydroxyindoleacetic acid levels. Hippocampal, but not midbrain or striatal, [3H]citalopram binding at 5-HT transporters was lower in WKY rats than in SHRs. However, neither [3H]5-HT reuptake kinetics nor the potencies of citalopram (1-1000 nM) to inhibit [3H]5-HT reuptake into hippocampal and striatal synaptosomes differed between strains. This was confirmed in vivo by means of microdialysis in the hippocampus of freely moving rats. Thus, although LEW rats displayed a 3-4 fold higher baseline level of extracellular 5-HT in the hippocampus, compared with SHRs and WKY rats, local perfusion with 1 microM citalopram promoted relative increases in extracellular 5-HT levels over baseline that were similar in all strains. Lastly, acute i.p. administration of 3.3 mg/kg citalopram (1 h beforehand) decreased to similar extents [3H]5-HT reuptake into hippocampal synaptosomes from SHRs and WKY rats. This study indicates that genetic differences in the behavioural responses to SSRIs may involve 5-HT transporter-independent mechanisms.  (+info)

The activity of rat brain nitric oxide synthase following chronic antidepressant treatment. (4/558)

Nitric oxide synthase (NOS) is an enzyme involved in the activation of the glutamate/NMDA receptor-induced cascade of events. In this study we investigated the NOS activity in different rat brain regions after chronic electroconvulsive, imipramine and citalopram treatments. Chronic electroconvulsive treatment significantly increased the NOS activity (by 49%) in the cerebral cortex. However, chronic treatment with imipramine or citalopram did not alter the activity of NOS in all examined brain regions (cortex, hippocampus or cerebellum). The increased NOS activity after electroconvulsive but not pharmacologic (imipramine or citalopram) treatment may well reflect the differences between the adaptive changes of the NMDA receptor complex induced by these treatments.  (+info)

Citalopram controls phobic symptoms in patients with panic disorder: randomized controlled trial. (5/558)

OBJECTIVE: To examine the effects of long-term treatment with citalopram or clomipramine on subjective phobic symptoms in patients with panic disorder. DESIGN: Double-blind, parallel-group, five-arm study. PATIENTS: Patients aged 18 to 65 years with panic disorder (DMS-III-R diagnosis) and with no major depressive symptoms. INTERVENTIONS: Four hundred and seventy-five patients were randomized to 8 weeks of treatment with either citalopram (10 to 15 mg per day; 20 to 30 mg per day; or 40 to 60 mg per day), clomipramine (60 to 90 mg per day) or placebo. Two hundred and seventy-nine patients continued treatment after the 8-week acute phase. OUTCOME MEASURES: Phobic symptoms were assessed using the Phobia Scale and the Symptom Checklist's (SCL-90) phobia-related factors. RESULTS: At all dosages, citalopram was more efficacious than placebo, with 20 to 30 mg generally being the most effective dosage. Citalopram (20 to 30 mg) generally decreased phobic symptoms significantly more than placebo after Month 3. Interpersonal sensitivity decreased when measured on the respective SCL-90 sub-scale. Alleviation of phobic symptoms generally continued to increase towards the end of the treatment. The effect of clomipramine was not as consistent. CONCLUSIONS: All active treatment groups, especially the group receiving 20 to 30 mg per day of citalopram, effectively controlled phobic symptoms in patients with panic disorder. Long-term treatment with citalopram further decreased phobic symptoms.  (+info)

Acute psychological effects of 3,4-methylenedioxymethamphetamine (MDMA, "Ecstasy") are attenuated by the serotonin uptake inhibitor citalopram. (6/558)

3,4-Methylenedioxymethamphetamine (MDMA, "Ecstasy") is a recreational drug that has been shown to release serotonin (5-HT) and dopamine (DA) in animals. The effect of MDMA on 5-HT release can be blocked by 5-HT uptake inhibitors such as citalopram, suggesting that MDMA interacts with the 5-HT uptake site. It is unknown whether this mechanism is also responsible for the psychological effects of MDMA in humans. We investigated the effect of citalopram pretreatment (40 mg iv) on the psychological effects of MDMA (1.5 mg/kg po) in a double-blind placebo-controlled psychometric study in 16 healthy human volunteers. MDMA produced an emotional state with heightened mood, increased self-confidence and extroversion, moderate derealization, and an intensification of sensory perception. Most of these effects were markedly reduced by citalopram. This finding suggests that the psychological effects of MDMA are mediated via action at the 5-HT uptake site to increase 5-HT release through the carrier, as expected from animal studies.  (+info)

Citalopram--a review of pharmacological and clinical effects. (7/558)

OBJECTIVE: To provide clinicians with a critical evaluation of citalopram, a selective serotonin reuptake inhibitor (SSRI) that has been available in Canada since March 1999. DATA SOURCES: Commercial searches (MEDLINE and BiblioTech) and an "in-house" search (InfoDrug) were used to find published English-language references for clinical and preclinical publications. There was no restriction of publication dates. Primary index terms used were: pharmacological properties, receptors, pharmacological selectivity, pharmacokinetics, age-related pharmacokinetics, sex-related pharmacokinetics, renal dysfunction, hepatic dysfunction, cytochrome activity, drug interactions, adverse reactions, antidepressant switching, precautions, overdose, drug discontinuation, children, geriatric, depression, combination therapy, placebo control, refractory depression, anxiety disorders and medical disorders. STUDY SELECTION: A total of 74 studies were reviewed. Twenty-one of these studies specifically examined the clinical efficacy and tolerability of citalopram in depressive disorders as well as other disorders. In depressive disorders, clinical studies were required to have either placebo or active comparison controls for a minimum of 3 weeks. For other disorders, in the absence of double-blind trials, open-label studies were included. Pharmacological studies were limited to animal studies focusing on citalopram's selectivity and receptor specificity, and positron emission tomography studies were incorporated to include human pharmacological data. Pharmacokinetic studies focused on the metabolism, safety and tolerability of citalopram, specifically with reference to adverse reactions, drug interactions and overdose in addition to citalopram's effect on vulnerable populations, such as children, the elderly and patients with metabolic diseases. DATA EXTRACTION: Data on clinical studies were summarized according to test measures, study duration and outcome of study. Pharmacokinetic and pharmacodynamic studies were summarized according to properties and interactions. Adverse reactions were extracted to outline citalopram's safety profile. DATA SYNTHESIS: Citalopram is an SSRI antidepressant with a more specific and selective pharmacological profile than other antidepressants of its class. It is well tolerated, and drug interactions are not a significant concern. It is also reasonably safe for populations vulnerable to pharmacokinetic effects, such as the elderly and patients with metabolic diseases. In addition to its tolerability, citalopram is effective in the treatment of major depression, other depressive disorders and panic disorder. It has the potential to effectively treat other anxiety disorders and substance-use disorders; in addition, it may be useful in several medical conditions. CONCLUSIONS: There is evidence to support the role of citalopram as a well-tolerated and effective SSRI antidepressant. There is a need for further evaluation of its role in psychiatric disorders other than major depressive disorder.  (+info)

Variations in response to citalopram in men and women with alcohol dependence. (8/558)

OBJECTIVE: To examine the differential effects of citalopram on alcohol consumption in nondepressed women and men with mild to moderate alcohol dependence. DESIGN: Prospective, placebo-controlled study. PARTICIPANTS: Sixty-one subjects (34 men and 27 women). INTERVENTIONS: After a 2-week baseline, subjects were randomly assigned to 12 weeks of citalopram (40 mg per day) (n = 15 women, 16 men) or placebo (n = 12 women, 18 men). All received brief standard psychosocial interventions. OUTCOME MEASURES: Alcohol Dependence Scale, Montgomery-Asberg Depression Scale, Michigan Alcohol Screening Test, State-Trait Anxiety Inventory and daily alcohol intake. RESULTS: Pretreatment sex differences were evident in alcohol consumption, alcohol dependence, alcohol-related problems and on anxiety and depression measures. After treatment, analyses of covariance with depression and anxiety scores as covariates revealed a differential benefit of citalopram for men. Men receiving citalopram reduced average drinks per day by 44%, whereas women exhibited a 27% decrease (p < 0.05). CONCLUSIONS: Men may benefit more than women from citalopram in the treatment of alcohol dependence. These findings highlight the importance of examining sex as a significant variable in evaluating response to pharmacotherapy.  (+info)

*Citalopram

... has been found to reduce the symptoms of diabetic neuropathy. While on its own citalopram is less effective than ... Treatment with citalopram should be reduced gradually when treatment is finished. Citalopram and other SSRIs can induce a mixed ... As with other SSRIs, citalopram can cause an increase in serum prolactin level. Citalopram has no significant effect on insulin ... Evidence for effectiveness of citalopram for treating depression in children is uncertain. Citalopram is licensed in the UK and ...

*Macropsia

Citalopram-induced macropsia is similar to zolpidem-induced macropsia since both types have been observed in relatively few ... Citalopram is an antidepressant that inhibits serotonin reuptake. The first case of macropsia thought to be induced by ... Ghanizadeh, A. Citalopram-induced macropsia. Clin Neuropharamcol 2007; 30(4): 246-247. Unnithan SB, Cutting JC. The cocaine ... Citalopram. A review of its pharmacodynamic and pharmacokinetic properties, and therapeutic potential in depressive illness. ...

*Brazilian Controlled Drugs and Substances Act

CITALOPRAM 26. CLOMACRANE 27. CLOMETIAZOLE 28. CLOMIPRAMINE 29. CLOREXADOL 30. CLORPROMAZINE 31. CHLOROPROXENE 32. CLOTIAPINE ...

*CYP2D6

"Citalopram Oral Solution". Drugs.com. Nevels RM, Weiss NH, Killebrew AE, Gontkovsky ST (July 2013). "Methylphenidate and Its ...

*List of drugs: T-Td

Redirects to citalopram. talopram (INN) talosalate (INN) talotrexin ammonium (USAN) taloximine (INN) talsaclidine (INN) ...

*Selective serotonin reuptake inhibitor

"FDA Drug Safety". Citalopram and escitalopram: QT interval prolongation-new maximum daily dose restrictions (including in ... The SSRIs, in decreasing toxicity in overdose, can be listed as follows: > citalopram (due to the potential for QT interval ... The recommended maximum daily dose of citalopram and escitalopram was reduced due to concerns with QT interval prolongation. In ... Oström M, Eriksson A, Thorson J, Spigset O (1996). "Fatal overdose with citalopram". Lancet. 348 (9023): 339-40. doi:10.1016/ ...

*Standard for the Uniform Scheduling of Medicines and Poisons

Citalopram), SNRIs (e.g. Duloxetine, Milnacipran), TCAs (e.g. Imipramine and MAOIs (e.g. Selegiline, Moclobemide). ...

*Impulsivity

Koran, Lorrin M.; Bullock, Kim D.; Hartston, Heidi J.; Elliott, Michael A.; d'Andrea, Vincent (2002). "Citalopram Treatment of ... Pharmacological interventions include the use of SSRIs, such as fluvoxamine, citalopram, escitalopram, and naltrexone Impulse ...

*Pseudobulbar affect

Andersen, G; Vestergaard, K; Riis, J. O. (1993). "Citalopram for post-stroke pathological crying". The Lancet. 342 (8875): 837- ... Traditionally, antidepressants such as sertraline, fluoxetine,citalopram, nortriptyline and amitriptyline have been prescribed ...

*Death of Conrad Roy

In the United States, citalopram carries a boxed warning stating it may increase suicidal thinking and behavior in those under ... "Celexa (citalopram hydrobromide) Tablets/Oral Solution" (pdf). Prescribing Information. Forest Laboratories, Inc. Press, The ...

*Escitalopram

Citalopram and escitalopram are associated with dose-dependent QT interval prolongation and should not be used in those with ... citalopram and venlafaxine in the treatment of major depression in Spain: clinical and economic consequences". Curr Med Res ... Whether escitalopram is more effective than citalopram or merely an effort to extend a patent over a product in which it was ... The most recent of these have concluded (with caveats in some cases) that escitalopram is modestly superior to citalopram in ...

*Development and discovery of SSRI drugs

... called citalopram. But citalopram can also be viewed as a constrained analogue of paroxetine. Citalopram has the second most ... Citalopram is composed of two enantiomers, (R)- and (S)-, which are mirror images of each other (figure 7). Researches has ... were able to generate a model of the (S)-citalopram binding site in human SERT by combining mutational analysis and comparative ... S)-citalopram is positioned as that the cyanophthalane-. fluorophenyl- and methylaminoprpyl moieties occupy three different sub ...

*Serotonin syndrome

Isbister GK, Dawson A, Whyte IM (September 2001). "Citalopram overdose, serotonin toxicity, or neuroleptic malignant syndrome ...

*Enantiomer

Citalopram is a racemate [1:1 mixture of (S)-citalopram and (R)-citalopram]; escitalopram [(S)-citalopram] is a pure enantiomer ... The dosages for escitalopram are typically 1/2 of those for citalopram. There are two main strategies for the preparation of ... Another example is the antidepressant drugs escitalopram and citalopram. ...

*Orally disintegrating tablet

Biovail Product Pipeline Citalopram Prescribing Information Ultracet Prescribing Information. ...

*Racemization

Jacquot, C.; David, D. J.; Gardier, A. M.; Sánchez, C. (2007). "Escitalopram and citalopram: the unexpected role of the R- ... Likewise, the (S) stereoisomer is much more reactive than the (R) enantiomer in citalopram (Celexa), an antidepressant which ...

*Reuptake inhibitor

Chen F, Larsen MB, Sánchez C, Wiborg O (2005). "The S-enantiomer of R,S-citalopram, increases inhibitor binding to the human ... A few of the selective serotonin reuptake inhibitors (SSRIs) such as the dextro-enantiomer of citalopram appear to be ... Ravna AW, Sylte I, Dahl SG (2003). "Molecular mechanism of citalopram and cocaine interactions with neurotransmitter ...

*GRIK4

A test of that gene can be made in order to know if a depressed patient will respond to the SSRI citalopram. The GRIK4 gene ... "GRIK4, HTR2A markers indicate reduced risk of nonresponse to citalopram". Science News. Science Daily. Retrieved 2009-01-20. ...

*Reuptake

exposed HEK-SERT with citalopram on a long-term basis. They noticed that long-term exposure led to a down-regulation of binding ... found that after removing citalopram from the system, normal levels of SERT binding site expression returned. Depression has ... they were able to quantify that citalopram acted on SERT as an SSRI, and that desipramine had no effect on SERT. In a separate ... They presented SERT with varying doses of either citalopram (an SSRI) or desipramine (an inhibitor of norepinephrine reuptake ...

*Cimetidine

"Pharmacokinetic interaction study of citalopram and cimetidine in healthy subjects". European Journal of Clinical Pharmacology ...

*Didesmethylcitalopram

... the S-enantiomer of citalopram. Like citalopram and escitalopram, didesmethyl(es)citalopram functions as a selective serotonin ... Didesmethylcitalopram is an active metabolite of the antidepressant drug citalopram (racemic). Didesmethylescitalopram is an ...

*Torsades de pointes

However, a study, "Evaluation of the FDA Warning Against Prescribing Citalopram at Doses Exceeding 40 mg," reported no ... 2012). "Prolonged QTc interval and torsades de pointes induced by citalopram". Tex. Heart Inst. J. 39 (1): 68-70. PMC 3298934 ... "FDA Drug Safety Communication: Revised recommendations for Celexa (citalopram hydrobromide) related to a potential risk of ... citalopram) above 40 mg per day, considered the maximum allowable dosage, thereby increasing the risk of Torsades. ...

*Serenic

... by citalopram". Neuropsychopharmacology. 33 (5): 1192-205. doi:10.1038/sj.npp.1301491. PMID 17609680. Dumont GJ, Sweep FC, van ...

*S100A10

"p11 Protein Levels in Patients With Major Depressive Disorder Treated With Citalopram". ClinicalTrials.gov. Akiba S, Hatazawa R ... and determine whether p11 levels in patients are affected by treatment with citalopram (Celexa), a serotonin reuptake inhibitor ...

*Bone morphogenetic protein 7

2010). "A genomewide association study of citalopram response in major depressive disorder". Biol. Psychiatry. 67 (2): 133-8. ...
We read with great interest the article by Tack et al on the effect of the selective serotonin reuptake inhibitor (SSRI) citalopram on symptoms in patients with irritable bowel syndrome (IBS) (Gut 2006;55:1095-103). The usefulness of the results of this study are however debatable. Several previous studies have investigated the effect of tricyclic antidepressants and SSRIs on functional gastrointestinal symptoms. Because of errors or lack of clarity in study design, inclusion of very selected patient populations and, above all, small sample sizes, their role in the treatment of patients with IBS in daily clinical practice remains unclear.. The study of Tack et al, as already correctly pointed out by Creed in his commentary (Gut 2006;55:1065-7), also suffers from major shortcomings in study design, poor description of study population and no information on whether or not subjects and physicians/investigators were blinded and, if yes, how ...
citalopram témoignage citalopram 20 citalopram danger citalopram vidal citalopram eureka. Citalopram HBr Tablet and placebo groups were compared with.Medical uses. Venlafaxine is used primarily for the treatment of depression, general anxiety disorder, social phobia, panic disorder, and vasomotor symptoms.. . naltrexone, (ReVia), fluoxetine (Prozac), citalopram (Celexa), paroxetene (Paxil),. compared with about 20% of Caucasians and fewer than 5% of African.compared with 1.5 points in the placebo-treated. Citalopram Selective serotonin reuptake inhibitor. Fluoxetine SSRI Depression Less sedating than citalopram,.citalopram 40 mg pills over the internet no prescription citalopram 40 mg pills. Wall contributing to the one that second generation, it the health of feel so bad...diminished response to antidepressants compared with younger females, there are also studies that conflict. ficacy of the TCA clomipramine, the SSRIs citalopram.. ...
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The researchers also measured depression and anxiety scores.. The effect of acute administration of citalopram on colonic sensitivity and on colonic response to feeding was assessed as a predictor of response to the drug.. The research team found after 3 and 6 weeks of treatment that citalopram improved abdominal pain, and bloating.. Impact of symptoms on daily life, and overall well being compared was also improved after 3 and 6 weeks with citalopram.. There was only a modest effect on stool pattern.. Changes in depression or anxiety scores were not related to symptom improvement.. The team noted that the effect of acute administration of citalopram during a colonic barostat study did not predict clinical outcome.. The researchers confirmed the benefit of citalopram over placebo after the analysis of the first treatment period as a double blind parallel arm study.. Dr Tacks team concluded, The selective serotonin reuptake inhibitors citalopram significantly improves irritable bowel syndrome ...
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NG Citalopram: Citalopram belongs to a group of medications called selective serotonin reuptake inhibitors (SSRIs). It is used to treat depression. SSRIs improve depression by increasing the amount of serotonin (a neurotransmitter) in certain areas of the brain, which in turn improves the ability of the brain to transmit messages from one nerve cell to another.
CO Citalopram: Citalopram belongs to a group of medications called selective serotonin reuptake inhibitors (SSRIs). It is used to treat depression. SSRIs improve depression by increasing the amount of serotonin (a neurotransmitter) in certain areas of the brain, which in turn improves the ability of the brain to transmit messages from one nerve cell to another.
... ! Citalopram belongs to a group of medications called selective serotonin reuptake inhibitors (SSRIs). Increased serotonin levels can help improve mood.
Citalopram is a selective serotonin reuptake inhibitor, or SSRI, that is commonly prescribed for the treatment of depression, according to WebMD. Citalopram is the generic version of Celexa and is...
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Objective:. Major depressive disorder (MDD) is a serious, debilitating, life-shortening illness that affects many persons of all ages and backgrounds. Although many patients suffering from MDD can expect improvement with antidepressant treatment, only a minority experience full remission, and little is known about the basis for individual differences in treatment outcome. The rationale for this study follows from the findings that treatment response in major depression is associated with: 1) variation in the genes encoding the serotonin 2A receptor (HTR2A) and the KA1 subunit of the glutamate-kainate receptor (GRIK4); and 2) increases in prefrontal glutamate/glutamine (glx) concentration measured by [+H] magnetic resonance spectroscopy (MRS). The central hypothesis is that genes associated with citalopram treatment outcome modulate glutamate concentrations in prefrontal brain (PFB) and thus facilitate response to citalopram treatment. Such information is important because it would ultimately ...
Early life stress is a potential precursor of eventual neuropsychiatric diseases and may result in altered neurodevelopment and function of the hippocampus, which thus provides a site at which potential interventions to modify the effects of early life stress may act. In this study, Sprague Dawley rat pups comprising male and female animals underwent maternal separation (MS) for 180 min from postnatal days (PND) 2 to 14, or were left with their dams. They subsequently received daily administration of saline (0.9%), escitalopram (10 mg/kg), or no treatment during adolescence (PND 43-60). All adult animals underwent brain magnetic resonance imaging (MRI) and bilateral hippocampal proton magnetic resonance spectroscopy ((1)H-MRS). Neither MS nor escitalopram treatment had a significant effect on hippocampal volume. Adult rats that experienced MS displayed significantly increased choline-containing compounds (Cho) and decreased N-acetylaspartate (NAA), glutamate (Glu) and Myo-inositol (MI) relative ...
Escitalopram is the first antidepressant introduced according to chirality rules, it is an S-enantiomer of citalopram, the drug which has been used for many years. Experimental studies showed that the property of serotonin transporter inhibition - one of the main mechanisms of antidepressive action is connected with the S-enantiomer of citalopram, and that escitalopram is the most selective inhibitor of this transporter. The results of most clinical studies in patients with depression show significant superiority of escitalopram, 10-20 mg/day, over placebo, as early as within the first week of treatment, and a faster onset of action and higher therapeutic efficacy of escitalopram, compared to citalopram, 20-40 mg/day. A similar efficacy of escitalopram, 10mg/day, and sertraline 50-200 mg/day, as well as escitalopram, 20 mg/day, and venlafaxine, 225 mg/day was demonstrated. It has also been shown that escitalopram, 10-20 mg/day, exerts therapeutic efficacy in general anxiety disorder, panic ...
Background: Subsyndromal symptoms of depression (SSD) in older outpatients with schizophrenia are common and clinically important. While many physicians prescribe antidepressants to patients with schizophrenia and schizoaffective disorder who have SSD, evidence for their effectiveness and safety has been meager. We describe a randomized placebo-controlled trial of citalopram in 198 patients.. Method: Participants in this 2-site study, conducted from September 1, 2001, to August 31, 2007, were men and women with DSM-IV schizophrenia or schizoaffective disorder who were 40 years of age or older and who met study criteria for SSD. Patients were randomly assigned to flexible-dose treatment with citalopram or placebo augmentation of their current antipsychotic medication. Analysis of covariance was used to compare improvement in scores on the Hamilton Rating Scale for Depression and Calgary Depression Rating Scale between treatment groups; secondary efficacy analyses compared improvement in several ...
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A simple and sensitive, solid-phase extraction method for the removal of Citalopram drug from waste water has been developed by using magnetic nanoparticles modified with surfactant sodium dodecyl sulfate. These magnetic nanoparticles have shown great adsorptive tendency towards Citalopram drug. The effect of different parameters influencing the extraction efficiency of this drug were investigated and optimized including the pH, amount of the surfactant, contact time and temperature. The extracts were analyzed by ultraviolet spectrophotometry at 239nm. Under these conditions, the related standard deviation (RSD %) of the method at two concentrations (5 and 50µg.mL-1) was in the range of (3.14â€
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Context The corticotropin-releasing factor (CRF, or corticotropin-releasing hormone) and arginine vasopressin systems have been implicated in the pathophysiology of anxiety and depressive disorders and response to antidepressant treatment.. Objective To study the association of genetic variants in 10 genes that regulate the CRF and arginine vasopressin systems with treatment response to citalopram in the Sequenced Treatment Alternatives to Relieve Depression (STAR*D) sample (N = 1768).. Design Pharmacogenetic association study derived from the STAR*D study, a multicenter, prospective, open, 12-week effectiveness trial.. Setting Outpatient primary care and psychiatric clinics.. Patients Individuals with nonpsychotic major depressive disorder for whom DNA was available who were subsequently treated with citalopram hydrobromide for 4 to 12 weeks.. Intervention Flexible doses of citalopram.. Main Outcome Measure Association of genetic polymorphisms in genes encoding the CRF system with response and ...
BACKGROUND: Although pharmacological and psychological interventions are both effective for major depression, antidepressant drugs remain the mainstay of treatment in primary and secondary care settings. During the last 20 years, antidepressant prescribing has risen dramatically in western countries, mainly because of the increasing consumption of selective serotonin reuptake inhibitors (SSRIs) and newer antidepressants, which have progressively become the most commonly prescribed antidepressants. Escitalopram is the pure S-enantiomer of the racemic citalopram. OBJECTIVES: To assess the evidence for the efficacy, acceptability and tolerability of escitalopram in comparison with tricyclics, other SSRIs, heterocyclics and newer agents in the acute-phase treatment of major depression. SEARCH STRATEGY: Electronic databases were searched up to July 2008. Trial databases of drug-approving agencies were hand-searched for published, unpublished and ongoing controlled trials. SELECTION CRITERIA: All randomised
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This is the business view business. Citalopram is an antidepressant in a group of drugs called selective serotonin reuptake inhibitors (SSRIs). It affects a naturally occuring chemical in the brain called serotonin, which helps to improve certain mood problems, but may become unbalanced and...
Citalopram is a selective serotonin reuptake inhibitor, balances levels of serotonin in the brain and heals major depression associated with mood disorders.
The US-based Sequenced Treatment Alternatives to Relieve Depression (STAR*D) project [17] enrolled a total of 4,041 treatment-seeking adults. All patients included in the study had a diagnosis of non-psychotic major depressive disorder (as confirmed by DSM-IV criteria), scoring greater than 14 on the HAM-D (that is, suffered from moderate to severe depression). Among the sample, there were high rates of chronic or recurrent depression. STAR*D had a multistep design; all patients received citalopram (an SSRI) in the first treatment level, then those without sufficient symptomatic benefit moved onto further treatment levels where they received either alternative or augmented treatment options.. STAR*D is the largest and longest study to ever be conducted evaluating antidepressant treatments. It was found that with first-level treatment with citalopram, approximately 30% of patients achieved remission. While the study was not expressly designed for pharmacogenetic purposes, DNA was collected from ...
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CIpralex seems not to have the same side effects as citalopram, its a nicer drug for me, doesn;t help me much as i am told should be expected for a borderline but is more tolerable than Cipralex is an updated version of Citalopram Escitalopram is an isomer of citalopram (so basically the same thing). Author: Flavio Guzman, MD Psychiatrist Pharmacology Department University of Mendoza Argentina. Citalopram (Celexa) and escitalopram (Lexapro) are two commonly prescribed SSRIs used for the treatment of depression and anxiety disorders. This brief multimedia tutorial explores the differences and similiarities ‎Introduction: Basic Concepts · ‎Pharmacology · ‎Indications and Off-Label. Hi everyone,I was fine wondering if cipralex,celexa,lexapro,are they the same med,or do they have any more differences.. ...
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Doctors give unbiased, trusted information on the benefits and side effects of Viibryd to treat Anxiety: Dr. Kumar on viibryd for anxiety: Citalopram and Buspirone are both described as being effective for anxiety. The magnitude of the difference between their effects is likely small if studied formally in a research setting although my experience with both is that i would predict someone would experience more comprehensive reduction in the severity of their anxiety and panic attacks in the context of citalopram treatment.
Citalopram is an antidepressant (selective serotonin reuptake inhibitor) and is used to treat depression. Learn about side effects, interactions and Maximum dose: 40 mg orally per day. Comments: The initial dose may be increased if necessary to 40 mg once a day after at least 1 week of therapy. In a single-dose study, citalopram AUC and half-life were increased in the subjects ≥ 60 years old by 30% and 50%, respectively, whereas in a multiple-dose study they were increased by 23% and 30%, respectively. 20 mg/day is the maximum recommended dose for patients who are greater than 60 years of age (see.. I have been on cit 20mg for 9 months and in therapy. Elsewhere anxiety and depression returned so my doc instructed my dosage to 30mg. On day 18 of new merchant. Anxiety is worse immediately in the morning. Cant graph the house. Cant eat and anything I do eat sweets a trip to the bathroom!. Short strange Increasing citalopram. Celexa 20 mg and 40 mg are fast-coated, celexa 30 mg tab, scored ...
Against this background, concern has been expressed during recent years that two of the newer serotonergic antidepressants, citalopram and venlafaxine, may possibly be more lethal in overdose than other newer compounds.. Concern over citalopram arose as a result of a report of six deaths following overdoses of this drug, raising the possibility that it could be intrinsically more toxic than other SSRIs (Ostromet al, 1996). However, no fatalities occurred among 44 people who took overdoses of citalopram alone in quantities ranging from 70 to 3000 mg (Personneet al, 1997). Widened QRS complexes in the electrocardiogram (ECG) were observed, and/or convulsions occurred, in about a third of individuals who had taken 600-1800 mg (30-90 × 20 mg tablets) and in all of those who had taken more than 1900 mg (95 × 20 mg tablets). Although there is uncertainty as to the cause of death in the citalopram fatalities, overdoses of other SSRIs were not associated with convulsions or ECG abnormalities to the ...
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All medicines may cause side effects, but many people have no, or minor, side effects.. Check with your doctor if any of these most common side effects persist or become bothersome:. Decreased sexual desire or ability; diarrhea; dizziness; drowsiness; dry mouth; increased sweating; lightheadedness when you stand or sit up; loss of appetite; nausea; stuffy nose; tiredness.. Seek medical attention right away if any of these severe side effects occur:. Severe allergic reactions (rash; hives; itching; difficulty breathing; tightness in the chest; swelling of the mouth, face, lips, or tongue); absent menstrual period; bizarre behavior; black or bloody stools; chest pain; confusion; decreased concentration; decreased coordination; fainting; fast or irregular heartbeat; hallucinations; memory loss; new or worsening agitation, panic attacks, aggressiveness, impulsiveness, irritability, hostility, exaggerated feeling of well-being, restlessness, or inability to sit still; persistent, painful erection; ...
Before you begin taking a medication, be sure to inform your doctor of any medical conditions or allergies you may have, any medications you are taking, whether you are pregnant or breast-feeding, and any other significant facts about your health. These factors may affect how you should use this medication.. Abnormal heart rhythm: Citalopram may cause an abnormal heart rhythm, especially at higher doses. Your doctor may occasionally monitor your heart rate and rhythm with a test called an electrocardiogram. People with a history of a heart rhythm disturbance called QT prolongation should not take this medication. If you have congestive heart failure, slow heart rhythm, are at risk of low potassium or magnesium levels because of certain illnesses or medications, or you are taking certain medications that can affect the heart rhythm (e.g., amiodarone, sotalol), discuss with your doctor how this medication may affect your medical condition, how your medical condition may affect the dosing and ...
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Changes in Depressive Symptoms and Social Functioning in the Sequenced Treatment Alternatives to Relieve Depression Study John W. Denninger, MD, PhD,* Adrienne O. van Nieuwenhuizen, MSc,* Stephen R. Wisniewski, PhD,Þ James F. Luther, MSc,Þ Madhukar H. Trivedi, MD,þ A. John Rush, MD,§ Jackie K. Gollan, PhD, Diego A. Pizzagalli, PhD,¶ and Maurizio Fava, MD* antidepressants, social functioning often does not return to baseline Abstract: Major depressive disorder (MDD) profoundly affects social func- (Weissman, 2000). A small number of studies have investigated long- tioning, including the ability to enjoy social activities with peers, friends, and term social functioning impairments during or after treatment using family members. We sought to compare changes in social functioning and antidepressants; these suggest that long-term impairments in social depressive symptoms in the first level of the Sequenced Treatment Alterna- functioning may persist even in patients whose depression is deter- ...
J Neurosurg 126:1598-1605, 2017. The pathophysiological mechanisms responsible for the formation of intracranial aneurysms (IAs) remain only partially elucidated. However, current evidence suggests a genetic component. The purpose of this study was to investigate the specific anatomical variations in the arterial complex that are associated with the presence of anterior communicating artery (ACoA) aneurysms in the familial forms of IAs.. METHODS This multicenter study investigated bifurcation IAs in patients who had a sporadic ACoA IA without a family history of IA (SACAA group), in patients who had an ACoA IA with a family history of IA (FACAA group), and in their healthy first-degree relatives (HFDRs). Through the use of MR angiography (MRA) reconstructions, the symmetry of the A1 segments and the angle between the A1 and A2 segments were analyzed on 3D models for each group. These measurements were then compared among the 3 groups.. RESULTS Twenty-four patients with SACAA, 24 patients with ...
BETHESDA, Md. -- The success of treatment with the antidepressant citalopram (Celexa) depends partly on a common genetic variation, researchers here said.
While the FDA has warned against use of the antidepressant citalopram at greater than 40 mg because of the risk for abnormal heart rhythms, a new study was unable to identify increased incidences of adverse cardiac events when the drug was administered at higher doses. Find out the study results and why the authors contend the FDA warning may be doing more harm than good. ...
Antidepressants. These medications influence the activity of brain chemicals (neurotransmitters) thought to play a role in anxiety disorders. Examples of antidepressants used to treat anxiety disorders include fluoxetine (Prozac, Sarafem), imipramine (Tofranil), paroxetine (Paxil, Pexeva), sertraline (Zoloft) and venlafaxine (Effexor XR). Citalopram (Celexa) and escitalopram (Lexapro) also can be effective, but dosages of 40 milligrams (mg) a day of citalopram or 20 mg a day of escitalopram warrant discussion of risks versus benefits. Your doctor also may recommend other antidepressants ...
How I wish Id known BEFORE all this happened what I know now, how I wish thered been more informed consent, more information, more care and that somehow Id have found my way to Surviving Antidepressants to understand and be forearmed. Thank you to SA for the information and support you give to so many. The "professionals" still seem to be ignoring any hint of these drugs causing damage (in my experience of them anyway). And I, naively, thought they were like taking an aspirin - more or less benign. Started Citalopram in 2006 (for depression) took it for a year, no noticeable side effects (but also a very subtle benefit, if there was one). 2007-2011 on and off Citalopram, some side effects when off it (first anxiety attack ever, hmmm, now I wonder if this was withdrawal). 2011 back on Citalopram because of anxious feelings, 2013 report to doctor that Im very tired all the time and yawning inappropriately. Doctor suggests trying another a-d. Late 2013-mid 2014, taken through a series of meds. ...
Pregnancy Category C. In a rat embryo/fetal development study, oral administration of escitalopram (56 mg/kg/day, 112 mg/kg/day, or 150 mg/kg/day) to pregnant animals during the period of organogenesis resulted in decreased fetal body weight and associated delays in ossification at the two higher doses (approximately ≥ 56 times the maximum recommended human dose [MRHD] of 20 mg/day on a body surface area [mg/m2] basis). Maternal toxicity (clinical signs and decreased body weight gain and food consumption), mild at 56 mg/kg/day, was present at all dose levels. The developmental no-effect dose of 56 mg/kg/day is approximately 28 times the MRHD on a mg/m2 basis. No teratogenicity was observed at any of the doses tested (as high as 75 times the MRHD on a mg/m2 basis).. When female rats were treated with escitalopram (6 mg/kg/day, 12 mg/kg/day, 24 mg/kg/day, or 48 mg/kg/day) during pregnancy and through weaning, slightly increased offspring mortality and growth retardation were noted at 48 ...
Escitalopram Generique Fiable Générique Lexapro Ou Achetez Peu Couteux Lexapro 10 mg. Lexapro (escitalopram) est un médicament antipsychotique appartenant à la classe des inhibiteurs sélectifs du recaptage de la sérotonine (ISRS) Il est utilisé pour traiter la dépression et le trouble danxiété généralisée (TAG). Il
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At 6 weeks, 11% of escitalopram group had stopped treatment versus 5% placebo (p=0.04). At 12 weeks, 15% of patients in escitalopram group discontinued treatment compared to 7% of placebo (p=0.02). Reasons for discontinuation were not reported. The only notable difference in safety parameters was worsening depression (9 patients in escitalopram versus 21 in placebo, p=0.03).. The authors concluded that escitalopram was safely tolerated in patients with HF and a low EF, however it did not reduce all-cause mortality, hospitalizations, or symptoms of depression. These results were similar to SAD HART-CHF. There was no identifiable benefit from treatment.. Key strengths of the MOOD-HF trial include the study design (randomized, double-blind, intention-to-treat analysis) with the concurrent use non-pharmacologic interventions in both study groups. The study duration was 18 months, significantly longer than SADHART-CHF. Additionally, monitoring of adherence using escitalopram serum levels (although ...
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Before you begin using a medication, be sure to inform your doctor of any medical conditions or allergies you may have, any medications you are taking, whether you are pregnant or breast-feeding, and any other significant facts about your health. These factors may affect how you should use this medication.. Abnormal heart rhythm: Escitalopram may cause an abnormal heart rhythm, especially at higher doses. Your doctor may occasionally monitor your heart rate and rhythm with a test called an electrocardiogram. People with a history of a heart rhythm disturbance called QT prolongation should not take this medication. If you have congestive heart failure, have slow heart rhythm, are at risk of low potassium or magnesium levels because of certain illnesses or medications, or are taking certain medications that can affect the heart rhythm (e.g., amiodarone, sotalol), you should discuss with your doctor how this medication may affect your medical condition, how your medical condition may affect the ...
Medscape - Depression-specific dosing for Lexapro (escitalopram), frequency-based adverse effects, comprehensive interactions, contraindications, pregnancy & lactation schedules, and cost information.
In this eMedTV segment, we continue the discussion on the side effects of escitalopram. This selection lists specific reactions that may occur during treatment, including those that can be serious and require immediate medical attention.
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We provide only general information about medications which does not cover all directions, possible drug integrations, or precautions. Information on the site cannot be used for self-treatment and self-diagnosis. Any specific instructions for a particular patient should be agreed with your health care adviser or doctor in charge of the case. We disclaim reliability of this information and mistakes it could contain. We are not responsible for any direct, indirect, special or other indirect damage as a result of any use of the information on this site and also for consequences of self-treatment. ...
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The antidepressant, which may take weeks to begin working,. Citalopram is part of a class of drugs called selective serotonin reuptake inhibitors,.5 going from citalopram to lexapro 6 does lexapro or zoloft cause more weight gain. 9 is it ok to take lexapro every other day 10 lexapro high dose side effects.Take citalopram at around the. to explain any part you do not understand. Take citalopram exactly. Celexa oral: Uses, Side Effects, Interactions, Pictures.Reversible cerebral vasoconstriction syndrome:. Reversible cerebral vasoconstriction syndrome in a. not to take Hydroxycut and citalopram,.can you take niacin with celexa Celexa make u sleepy, i like celexa better than lexapro Celexa no prescription medicine. Citalopram is any dairy product can cause.. Community Experts online right now. Ask for FREE. Ask Your Question Fast!.What You Need to Know about Citalopram Withdrawal. When taking anti-depressants such as Citalopram, patients must not discontinue use without the advice of a ...
Definition of selective serotonin reuptake inhibitor in the Legal Dictionary - by Free online English dictionary and encyclopedia. What is selective serotonin reuptake inhibitor? Meaning of selective serotonin reuptake inhibitor as a legal term. What does selective serotonin reuptake inhibitor mean in law?
TY - JOUR. T1 - Use of benzodiazepines and selective serotonin reuptake inhibitors in middle-aged and older adults with anxiety disorders. T2 - A longitudinal and prospective study. AU - Benítez, Carlos Israel Pérez. AU - Smith, Kevin. AU - Vasile, Russell G.. AU - Rende, Richard. AU - Edelen, Maria Orlando. AU - Keller, Martin B.. PY - 2008/1. Y1 - 2008/1. N2 - Objective: The purpose of this study was to examine the use of benzodiazepines (BZs) and selective serotonin reuptake inhibitors/selective norepinephrine reuptake inhibitors (SSRIs/SNRIs) over nine years of follow-up in middle-aged and older adults with diagnoses of panic disorder with or without agoraphobia, social phobia, or generalized anxiety disorder. Setting and Participants: Participants in this study were enrolled in the Harvard/Brown Anxiety Research Project (HARP). HARP is a naturalistic, longitudinal, multisite study of adults with anxiety disorders who are recruited from psychiatric settings. The analytic sample consisted ...
To test the hypothesis that selective serotonin reuptake inhibitor (SSRI) antidepressants may have a suicide emergent effect, particularly in children and adolescents. Detections of different antidepressants in the forensic toxicological screening of 14 857 suicides were compared with those in 26 422 cases of deaths by accident or natural causes in Sweden 1992-2000. There were 3411 detections of antidepressants in the suicides and 1538 in the controls. SSRIs had lower odds ratios than the other antidepressants. In the 52 suicides under 15 years, no SSRIs were detected. In 15-19-year age group, SSRIs had lower relative risk in suicides compared with non-SSRIs. The hypothesis that treatment of depressed individuals with SSRIs leads to an increased risk of suicide was not supported by this analysis of the total suicidal outcome of the nationwide use of SSRIs in Sweden over a period of 9 years, either in adults or in children or adolescents.. ...
TY - JOUR. T1 - 2,5-Disubstituted tetrahydrofurans as selective serotonin re-uptake inhibitors. AU - Voelker, Troy. AU - Xia, Haiji. AU - Fandrick, Keith. AU - Johnson, Robert. AU - Janowsky, Aaron. AU - Cashman, John R.. PY - 2009/3/1. Y1 - 2009/3/1. N2 - Enhancement of 5-hydroxytryptamine (5-HT, serotonin) neurotransmission is a viable means of treating depression. On the basis of this observation, agents that inhibit re-uptake of 5-HT were prepared based on (-)-cocaine and aryltropanes as lead compounds because they are reasonably potent 5-HT re-uptake inhibitors. Molecular dissection of an aryltropane provided a series of 5- and 6-membered ring compounds. From among this library of compounds a series of disubstituted tetrahydrofurans bearing 2-alkyl aryl and 5-alkyl amino groups were identified as having highly potent and selective 5-HT re-uptake inhibition. The compounds were evaluated for their ability to compete with radiolabeled RTI-55 binding and to inhibit re-uptake of ...
Selective serotonin reuptake inhibitors (SSRIs): Alaproclate • Citalopram • Dapoxetine • Desmethylcitalopram • Desmethylsertraline • Escitalopram • Femoxetine • Fluoxetine • Fluvoxamine • Indalpine • Ifoxetine • Litoxetine • Lu AA21004 • Lubazodone • Panuramine • Paroxetine • Pirandamine • RTI-353 • Seproxetine • Sertraline • Vilazodone • Zimelidine; Serotonin-norepinephrine reuptake inhibitors (SNRIs): Bicifadine • Desvenlafaxine • Duloxetine • Eclanamine • Levomilnacipran • Milnacipran • Sibutramine • Venlafaxine; Serotonin-norepinephrine-dopamine reuptake inhibitors (SNDRIs): Brasofensine • Diclofensine • DOV-102,677 • DOV-21,947 • DOV-216,303 • NS-2359 • SEP-225289 • SEP-227,162 • Tesofensine; Tricyclic antidepressants (TCAs): Amitriptyline • Butriptyline • Cianopramine • Clomipramine • Desipramine • Dosulepin • Doxepin • Imipramine • Lofepramine • Nortriptyline • Pipofezine • Protriptyline • ...
Purpose To describe the risk of early- and late-onset preeclampsia across pregnancies exposed to antidepressants and to evaluate the impact of timing and length of gestational exposure to antidepressants, particularly selective serotonin reuptake inhibitors (SSRIs), on preeclampsia. Methods The Norwegian Mother and Child Cohort, a prospective population-based study, and the Medical Birth Registry of Norway provided information on antidepressant exposure, depression, and anxiety symptoms in pregnancy, preeclampsia diagnoses, and important covariates. Within a pregnancy cohort of depressed women, we compared the risk of late-onset preeclampsia between SSRI-exposed and nonmedicated pregnancies using marginal structural models (weighted) and modified Poisson regression models. Results Of the 5887 pregnancies included, 11.1% were exposed at any time before week 34 to SSRIs, 1.3% to serotonin-norepinephrine reuptake inhibitors, 0.4% to tricyclic antidepressants, and 0.5% to other antidepressants. The ...
Boursi B, Lurie I, Haynes K, Mamtani R, Yang Y. Chronic therapy with selective serotonin reuptake inhibitors and survival in newly diagnosed cancer patients, European Journal of Cancer Care, March 2017 [Epub ahead of print] doi: 10.1111/ecc.12666 Feffer K, Lapidus K, Braw Y, Bloch Y, Kron S, Netzer R, Nitzan U. Factors associated with response after Deep Transcranial Magnetic Stimulation in a real-world clinical setting: Results from the first 40 cases of treatment-resistant depression. European Psychiatry, July 2017; 61-67. Feffer K, Lichtenberg P, Becker G, Bloch Y, Netzer R, Nitzan U. A comparative study with depressed patients on the acceptability of placebo use. General Hospital Psychiatry, 2016, 41:53-6. doi: 10.1016/j.genhosppsych.2016.05.004.. Gvirts HZ, Mayseless N, Segev A, Lewis DY, Feffer K, Barnea Y, Bloch Y, Shamay-Tsoory SG. Novelty-seeking trait predicts the effect of methylphenidate on creativity. Journal of Psychopharmacology. 2017 May; 31(5): 599-605. doi: ...
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Citalopram: Pediatric Medication | Memorial Sloan Kettering Cancer CenterCitalopram: Pediatric Medication | Memorial Sloan Kettering Cancer Center

This information should not be used to decide whether or not to take this medicine or any other medicine. Only the healthcare provider has the knowledge and training to decide which medicines are right for a specific patient. This information does not endorse any medicine as safe, effective, or approved for treating any patient or health condition. This is only a brief summary of general information about this medicine. It does NOT include all information about the possible uses, directions, warnings, precautions, interactions, adverse effects, or risks that may apply to this medicine. This information is not specific medical advice and does not replace information you receive from the healthcare provider. You must talk with the healthcare provider for complete information about the risks and benefits of using this medicine.. ...
more infohttps://www.mskcc.org/cancer-care/patient-education/citalopram-01

Citalopram (GenRx) | healthdirectCitalopram (GenRx) | healthdirect

On this page about Citalopram (GenRx) you will find information relating to side effects, age restrictions, food interactions, ... Brand name: Citalopram (GenRx). Citalopram (GenRx) is a medicine containing the active ingredient(s) citalopram. On this page ... Active ingredient in this medicine: citalopram. No information has been found for the selected product. Contact your pharmacist ... you will find out more about Citalopram (GenRx), including side effects, age restrictions, food interactions and whether the ...
more infohttps://www.healthdirect.gov.au/medicines/brand/amt,3295011000036109/citalopram-genrx

PatientsLikeMe | Citalopram report for  patients like youPatientsLikeMe | Citalopram report for patients like you

Find the most comprehensive real-world treatment information on Citalopram at PatientsLikeMe. 1734 patients with fibromyalgia, ... bipolar I disorder or psoriasis currently take Citalopram. ... Reddys Lab Citalopram Co-Citalopram Seropram Talohexal Citabax ... What is Citalopram?. Category: Prescription Drugs Most popular types: Celexa Cipramil APO-Citalopram Citopam Cilift Novo- ... What are people saying about Citalopram?. There are 25 topics in our forum about Citalopram. ...
more infohttps://www.patientslikeme.com/treatments/show/390-citalopram-side-effects-and-efficacy?brand=false
  • Do not stop taking citalopram without talking to your doctor. (medlineplus.gov)
  • If it becomes necessary for you to stop taking citalopram, ask your doctor to advise you how best to gradually reduce the dose to avoid withdrawal effects. (pharmasave.com)
  • Levomethadyl, a medication used to treat opioid dependence, may cause toxicity to the heart if used together with citalopram. (encyclopedia.com)
  • Children younger than 18 years of age should not normally take citalopram, but in some cases, a doctor may decide that citalopram is the best medication to treat a child's condition. (medlineplus.gov)
  • The doctor or pharmacist will give you the manufacturer's patient information sheet (Medication Guide) when you begin treatment with citalopram. (medlineplus.gov)
  • Citalopram Start Up Affects? (drugs.com)
  • Citalopram - Do initial side affects go away? (drugs.com)
  • Do not engage in activities that require you to be alert until you know how citalopram affects you. (everydayhealth.com)
  • Some data suggest the effectiveness of intravenous infusion of citalopram in resistant OCD. (wikipedia.org)
  • The FDA also issued a Safety Announcement in 2012 recommending that citalopram not be prescribed at doses greater than 40 milligrams a day because of the increased risk potentially dangerous abnormalities in the electrical activity of the heart (changes in heart rhythm). (everydayhealth.com)
  • There is no clinical data available on the use of citalopram in children and adolescents. (encyclopedia.com)
  • Anyone considering the use of citalopram tablets or any other antidepressant in a child, adolescent, or young adult must balance this risk with the clinical need. (nih.gov)
  • Citalopram comes as a tablet and a solution (liquid) to take by mouth. (medlineplus.gov)
  • 1 tablet (filmovertrukken) indeholder 10 mg eller 30 mg (delekærv) citalopram. (medicin.dk)
  • Tolerance to the inhibition of 5-HT uptake is not induced by long-term (14-day) treatment of rats with citalopram. (nih.gov)
  • Citalopram enhances cocaine's subjective effects in rats. (nih.gov)
  • This study investigated the effects of citalopram on cocaine's discriminative-stimulus effects in rats trained more simply to discriminate cocaine from saline. (nih.gov)
  • Each symbol represents the average of six to ten rats except at 0.3 mg/kg cocaine (n = 4), 10 mg/kg citalopram and 0.3 mg/kg of cocaine (n=4), and 10 mg/kg citalopram and 10 mg/kg cocaine (n=3). (nih.gov)
  • For brands that may still be available, search under citalopram. (pharmasave.com)
  • The present findings suggest that enhancement of cocaine's discriminative-stimulus effects may occur through a mechanism different from that underlying enhancement of cocaine's locomotor effects or that another action of citalopram selectively blocks locomotor enhancement. (nih.gov)
  • Certain antifungal medications such as itraconazole, fluconazole, ketoconazole, as well as the antibiotic erythromycin, can increase the levels of citalopram in the body. (encyclopedia.com)
  • What if I start from flux and drink it 2months and if it not working good start with citalopram ?or it's dangerous to change from one to other? (healingwell.com)
  • There are studies suggesting that citalopram can be useful in reducing aggressive and impulsive behavior. (wikipedia.org)
  • Citalopram (GenRx) is a medicine containing the active ingredient(s) citalopram. (healthdirect.gov.au)
  • The name of your medicine is Chemmart Citalopram. (nps.org.au)
  • Other conditions that may be a concern when taking citalopram are a history of bipolar or manic-depressive disorder, past suicide attempts, liver disease, seizures, low blood sodium, intestinal ulcers or bleeding problems. (everydayhealth.com)