Ciprofloxacin: A broad-spectrum antimicrobial carboxyfluoroquinoline.Anti-Infective Agents: Substances that prevent infectious agents or organisms from spreading or kill infectious agents in order to prevent the spread of infection.Fluoroquinolones: A group of QUINOLONES with at least one fluorine atom and a piperazinyl group.Microbial Sensitivity Tests: Any tests that demonstrate the relative efficacy of different chemotherapeutic agents against specific microorganisms (i.e., bacteria, fungi, viruses).Anti-Bacterial Agents: Substances that reduce the growth or reproduction of BACTERIA.Quinolones: A group of derivatives of naphthyridine carboxylic acid, quinoline carboxylic acid, or NALIDIXIC ACID.Ofloxacin: A synthetic fluoroquinolone antibacterial agent that inhibits the supercoiling activity of bacterial DNA GYRASE, halting DNA REPLICATION.Drug Resistance, Bacterial: The ability of bacteria to resist or to become tolerant to chemotherapeutic agents, antimicrobial agents, or antibiotics. This resistance may be acquired through gene mutation or foreign DNA in transmissible plasmids (R FACTORS).DNA Gyrase: A bacterial DNA topoisomerase II that catalyzes ATP-dependent breakage of both strands of DNA, passage of the unbroken strands through the breaks, and rejoining of the broken strands. Gyrase binds to DNA as a heterotetramer consisting of two A and two B subunits. In the presence of ATP, gyrase is able to convert the relaxed circular DNA duplex into a superhelix. In the absence of ATP, supercoiled DNA is relaxed by DNA gyrase.Levofloxacin: The L-isomer of Ofloxacin.QuinolinesDrug Resistance, Microbial: The ability of microorganisms, especially bacteria, to resist or to become tolerant to chemotherapeutic agents, antimicrobial agents, or antibiotics. This resistance may be acquired through gene mutation or foreign DNA in transmissible plasmids (R FACTORS).Norfloxacin: A synthetic fluoroquinolone (FLUOROQUINOLONES) with broad-spectrum antibacterial activity against most gram-negative and gram-positive bacteria. Norfloxacin inhibits bacterial DNA GYRASE.DNA Topoisomerase IV: A bacterial DNA topoisomerase II that catalyzes ATP-dependent breakage of both strands of DNA, passage of the unbroken strands through the breaks, and rejoining of the broken strands. Topoisomerase IV binds to DNA as a heterotetramer consisting 2 parC and 2 parE subunits. Topoisomerase IV is a decatenating enzyme that resolves interlinked daughter chromosomes following DNA replication.Nalidixic Acid: A synthetic 1,8-naphthyridine antimicrobial agent with a limited bacteriocidal spectrum. It is an inhibitor of the A subunit of bacterial DNA GYRASE.Aza CompoundsPefloxacin: A synthetic broad-spectrum fluoroquinolone antibacterial agent active against most gram-negative and gram-positive bacteria.4-Quinolones: QUINOLONES containing a 4-oxo (a carbonyl in the para position to the nitrogen). They inhibit the A subunit of DNA GYRASE and are used as antimicrobials. Second generation 4-quinolones are also substituted with a 1-piperazinyl group at the 7-position and a fluorine at the 6-position.Drug Resistance, Multiple, Bacterial: The ability of bacteria to resist or to become tolerant to several structurally and functionally distinct drugs simultaneously. This resistance may be acquired through gene mutation or foreign DNA in transmissible plasmids (R FACTORS).Pseudomonas aeruginosa: A species of gram-negative, aerobic, rod-shaped bacteria commonly isolated from clinical specimens (wound, burn, and urinary tract infections). It is also found widely distributed in soil and water. P. aeruginosa is a major agent of nosocomial infection.Enoxacin: A broad-spectrum 6-fluoronaphthyridinone antibacterial agent that is structurally related to NALIDIXIC ACID.Fleroxacin: A broad-spectrum antimicrobial fluoroquinolone. The drug strongly inhibits the DNA-supercoiling activity of DNA GYRASE.Gram-Negative Bacteria: Bacteria which lose crystal violet stain but are stained pink when treated by Gram's method.Staphylococcus aureus: Potentially pathogenic bacteria found in nasal membranes, skin, hair follicles, and perineum of warm-blooded animals. They may cause a wide range of infections and intoxications.Pseudomonas Infections: Infections with bacteria of the genus PSEUDOMONAS.Gram-Positive Bacteria: Bacteria which retain the crystal violet stain when treated by Gram's method.Gram-Negative Bacterial Infections: Infections caused by bacteria that show up as pink (negative) when treated by the gram-staining method.DNA Topoisomerases, Type II: DNA TOPOISOMERASES that catalyze ATP-dependent breakage of both strands of DNA, passage of the unbroken strands through the breaks, and rejoining of the broken strands. These enzymes bring about relaxation of the supercoiled DNA and resolution of a knotted circular DNA duplex.Ceftriaxone: A broad-spectrum cephalosporin antibiotic with a very long half-life and high penetrability to meninges, eyes and inner ears.Salmonella paratyphi A: A serotype of SALMONELLA ENTERICA that causes mild PARATYPHOID FEVER in humans.Administration, Oral: The giving of drugs, chemicals, or other substances by mouth.Ceftazidime: Semisynthetic, broad-spectrum antibacterial derived from CEPHALORIDINE and used especially for Pseudomonas and other gram-negative infections in debilitated patients.Streptococcus pneumoniae: A gram-positive organism found in the upper respiratory tract, inflammatory exudates, and various body fluids of normal and/or diseased humans and, rarely, domestic animals.Gonorrhea: Acute infectious disease characterized by primary invasion of the urogenital tract. The etiologic agent, NEISSERIA GONORRHOEAE, was isolated by Neisser in 1879.Neisseria gonorrhoeae: A species of gram-negative, aerobic bacteria primarily found in purulent venereal discharges. It is the causative agent of GONORRHEA.Salmonella typhi: A serotype of SALMONELLA ENTERICA which is the etiologic agent of TYPHOID FEVER.Typhoid Fever: An acute systemic febrile infection caused by SALMONELLA TYPHI, a serotype of SALMONELLA ENTERICA.Colony Count, Microbial: Enumeration by direct count of viable, isolated bacterial, archaeal, or fungal CELLS or SPORES capable of growth on solid CULTURE MEDIA. The method is used routinely by environmental microbiologists for quantifying organisms in AIR; FOOD; and WATER; by clinicians for measuring patients' microbial load; and in antimicrobial drug testing.Bacteria, AerobicUrinary Tract Infections: Inflammatory responses of the epithelium of the URINARY TRACT to microbial invasions. They are often bacterial infections with associated BACTERIURIA and PYURIA.Bacterial Infections: Infections by bacteria, general or unspecified.Bacteria, AnaerobicEscherichia coli: A species of gram-negative, facultatively anaerobic, rod-shaped bacteria (GRAM-NEGATIVE FACULTATIVELY ANAEROBIC RODS) commonly found in the lower part of the intestine of warm-blooded animals. It is usually nonpathogenic, but some strains are known to produce DIARRHEA and pyogenic infections. Pathogenic strains (virotypes) are classified by their specific pathogenic mechanisms such as toxins (ENTEROTOXIGENIC ESCHERICHIA COLI), etc.Drug Therapy, Combination: Therapy with two or more separate preparations given for a combined effect.Gentamicins: A complex of closely related aminoglycosides obtained from MICROMONOSPORA purpurea and related species. They are broad-spectrum antibiotics, but may cause ear and kidney damage. They act to inhibit PROTEIN BIOSYNTHESIS.Rifampin: A semisynthetic antibiotic produced from Streptomyces mediterranei. It has a broad antibacterial spectrum, including activity against several forms of Mycobacterium. In susceptible organisms it inhibits DNA-dependent RNA polymerase activity by forming a stable complex with the enzyme. It thus suppresses the initiation of RNA synthesis. Rifampin is bactericidal, and acts on both intracellular and extracellular organisms. (From Gilman et al., Goodman and Gilman's The Pharmacological Basis of Therapeutics, 9th ed, p1160)Trimethoprim-Sulfamethoxazole Combination: This drug combination has proved to be an effective therapeutic agent with broad-spectrum antibacterial activity against both gram-positive and gram-negative organisms. It is effective in the treatment of many infections, including PNEUMOCYSTIS PNEUMONIA in AIDS.Paratyphoid Fever: A prolonged febrile illness commonly caused by several Paratyphi serotypes of SALMONELLA ENTERICA. It is similar to TYPHOID FEVER but less severe.Imipenem: Semisynthetic thienamycin that has a wide spectrum of antibacterial activity against gram-negative and gram-positive aerobic and anaerobic bacteria, including many multiresistant strains. It is stable to beta-lactamases. Clinical studies have demonstrated high efficacy in the treatment of infections of various body systems. Its effectiveness is enhanced when it is administered in combination with CILASTATIN, a renal dipeptidase inhibitor.Amikacin: A broad-spectrum antibiotic derived from KANAMYCIN. It is reno- and oto-toxic like the other aminoglycoside antibiotics.Eye Infections, Bacterial: Infections in the inner or external eye caused by microorganisms belonging to several families of bacteria. Some of the more common genera found are Haemophilus, Neisseria, Staphylococcus, Streptococcus, and Chlamydia.Bacteremia: The presence of viable bacteria circulating in the blood. Fever, chills, tachycardia, and tachypnea are common acute manifestations of bacteremia. The majority of cases are seen in already hospitalized patients, most of whom have underlying diseases or procedures which render their bloodstreams susceptible to invasion.Azithromycin: A semi-synthetic macrolide antibiotic structurally related to ERYTHROMYCIN. It has been used in the treatment of Mycobacterium avium intracellulare infections, toxoplasmosis, and cryptosporidiosis.Staphylococcal Infections: Infections with bacteria of the genus STAPHYLOCOCCUS.Half-Life: The time it takes for a substance (drug, radioactive nuclide, or other) to lose half of its pharmacologic, physiologic, or radiologic activity.Serum Bactericidal Test: Method of measuring the bactericidal activity contained in a patient's serum as a result of antimicrobial therapy. It is used to monitor the therapy in BACTERIAL ENDOCARDITIS; OSTEOMYELITIS and other serious bacterial infections. As commonly performed, the test is a variation of the broth dilution test. This test needs to be distinguished from testing of the naturally occurring BLOOD BACTERICIDAL ACTIVITY.Ampicillin: Semi-synthetic derivative of penicillin that functions as an orally active broad-spectrum antibiotic.Klebsiella pneumoniae: Gram-negative, non-motile, capsulated, gas-producing rods found widely in nature and associated with urinary and respiratory infections in humans.Escherichia coli Infections: Infections with bacteria of the species ESCHERICHIA COLI.Area Under Curve: A statistical means of summarizing information from a series of measurements on one individual. It is frequently used in clinical pharmacology where the AUC from serum levels can be interpreted as the total uptake of whatever has been administered. As a plot of the concentration of a drug against time, after a single dose of medicine, producing a standard shape curve, it is a means of comparing the bioavailability of the same drug made by different companies. (From Winslade, Dictionary of Clinical Research, 1992)Salmonella Infections: Infections with bacteria of the genus SALMONELLA.Cefuroxime: Broad-spectrum cephalosporin antibiotic resistant to beta-lactamase. It has been proposed for infections with gram-negative and gram-positive organisms, GONORRHEA, and HAEMOPHILUS.Cefotaxime: Semisynthetic broad-spectrum cephalosporin.Cephalosporins: A group of broad-spectrum antibiotics first isolated from the Mediterranean fungus ACREMONIUM. They contain the beta-lactam moiety thia-azabicyclo-octenecarboxylic acid also called 7-aminocephalosporanic acid.Enterobacteriaceae: A family of gram-negative, facultatively anaerobic, rod-shaped bacteria that do not form endospores. Its organisms are distributed worldwide with some being saprophytes and others being plant and animal parasites. Many species are of considerable economic importance due to their pathogenic effects on agriculture and livestock.Nitrofurantoin: A urinary anti-infective agent effective against most gram-positive and gram-negative organisms. Although sulfonamides and antibiotics are usually the agents of choice for urinary tract infections, nitrofurantoin is widely used for prophylaxis and long-term suppression.Piperacillin: Semisynthetic, broad-spectrum, AMPICILLIN derived ureidopenicillin antibiotic proposed for PSEUDOMONAS infections. It is also used in combination with other antibiotics.Azlocillin: A semisynthetic ampicillin-derived acylureido penicillin.Topoisomerase II Inhibitors: Compounds that inhibit the activity of DNA TOPOISOMERASE II. Included in this category are a variety of ANTINEOPLASTIC AGENTS which target the eukaryotic form of topoisomerase II and ANTIBACTERIAL AGENTS which target the prokaryotic form of topoisomerase II.Thienamycins: Beta-lactam antibiotics that differ from PENICILLINS in having the thiazolidine sulfur atom replaced by carbon, the sulfur then becoming the first atom in the side chain. They are unstable chemically, but have a very broad antibacterial spectrum. Thienamycin and its more stable derivatives are proposed for use in combinations with enzyme inhibitors.Drug Interactions: The action of a drug that may affect the activity, metabolism, or toxicity of another drug.Penicillin Resistance: Nonsusceptibility of an organism to the action of penicillins.Bacteria: One of the three domains of life (the others being Eukarya and ARCHAEA), also called Eubacteria. They are unicellular prokaryotic microorganisms which generally possess rigid cell walls, multiply by cell division, and exhibit three principal forms: round or coccal, rodlike or bacillary, and spiral or spirochetal. Bacteria can be classified by their response to OXYGEN: aerobic, anaerobic, or facultatively anaerobic; by the mode by which they obtain their energy: chemotrophy (via chemical reaction) or PHOTOTROPHY (via light reaction); for chemotrophs by their source of chemical energy: CHEMOLITHOTROPHY (from inorganic compounds) or chemoorganotrophy (from organic compounds); and by their source for CARBON; NITROGEN; etc.; HETEROTROPHY (from organic sources) or AUTOTROPHY (from CARBON DIOXIDE). They can also be classified by whether or not they stain (based on the structure of their CELL WALLS) with CRYSTAL VIOLET dye: gram-negative or gram-positive.Erythromycin: A bacteriostatic antibiotic macrolide produced by Streptomyces erythreus. Erythromycin A is considered its major active component. In sensitive organisms, it inhibits protein synthesis by binding to 50S ribosomal subunits. This binding process inhibits peptidyl transferase activity and interferes with translocation of amino acids during translation and assembly of proteins.DNA, Bacterial: Deoxyribonucleic acid that makes up the genetic material of bacteria.Trimethoprim: A pyrimidine inhibitor of dihydrofolate reductase, it is an antibacterial related to PYRIMETHAMINE. It is potentiated by SULFONAMIDES and the TRIMETHOPRIM, SULFAMETHOXAZOLE DRUG COMBINATION is the form most often used. It is sometimes used alone as an antimalarial. TRIMETHOPRIM RESISTANCE has been reported.Dry Powder Inhalers: A device that delivers medication to the lungs in the form of a dry powder.Mutation: Any detectable and heritable change in the genetic material that causes a change in the GENOTYPE and which is transmitted to daughter cells and to succeeding generations.Penicillins: A group of antibiotics that contain 6-aminopenicillanic acid with a side chain attached to the 6-amino group. The penicillin nucleus is the chief structural requirement for biological activity. The side-chain structure determines many of the antibacterial and pharmacological characteristics. (Goodman and Gilman's The Pharmacological Basis of Therapeutics, 8th ed, p1065)Vancomycin: Antibacterial obtained from Streptomyces orientalis. It is a glycopeptide related to RISTOCETIN that inhibits bacterial cell wall assembly and is toxic to kidneys and the inner ear.Keratitis: Inflammation of the cornea.Doxycycline: A synthetic tetracycline derivative with similar antimicrobial activity.Anti-Infective Agents, Urinary: Substances capable of killing agents causing urinary tract infections or of preventing them from spreading.Cross Infection: Any infection which a patient contracts in a health-care institution.Tobramycin: An aminoglycoside, broad-spectrum antibiotic produced by Streptomyces tenebrarius. It is effective against gram-negative bacteria, especially the PSEUDOMONAS species. It is a 10% component of the antibiotic complex, NEBRAMYCIN, produced by the same species.Tetracycline: A naphthacene antibiotic that inhibits AMINO ACYL TRNA binding during protein synthesis.Injections, Intravenous: Injections made into a vein for therapeutic or experimental purposes.Gram-Positive Bacterial Infections: Infections caused by bacteria that retain the crystal violet stain (positive) when treated by the gram-staining method.Methicillin Resistance: Non-susceptibility of a microbe to the action of METHICILLIN, a semi-synthetic penicillin derivative.Cilastatin: A renal dehydropeptidase-I and leukotriene D4 dipeptidase inhibitor. Since the antibiotic, IMIPENEM, is hydrolyzed by dehydropeptidase-I, which resides in the brush border of the renal tubule, cilastatin is administered with imipenem to increase its effectiveness. The drug also inhibits the metabolism of leukotriene D4 to leukotriene E4.Campylobacter jejuni: A species of bacteria that resemble small tightly coiled spirals. Its organisms are known to cause abortion in sheep and fever and enteritis in man and may be associated with enteric diseases of calves, lambs, and other animals.Mycobacterium fortuitum: A rapid-growing, nonphotochromogenic species that is potentially pathogenic, producing lesions of lung, bone, or soft tissue following trauma. It has been found in soil and in injection sites of humans, cattle, and cold-blooded animals. (Dorland, 28th ed)Metronidazole: A nitroimidazole used to treat AMEBIASIS; VAGINITIS; TRICHOMONAS INFECTIONS; GIARDIASIS; ANAEROBIC BACTERIA; and TREPONEMAL INFECTIONS. It has also been proposed as a radiation sensitizer for hypoxic cells. According to the Fourth Annual Report on Carcinogens (NTP 85-002, 1985, p133), this substance may reasonably be anticipated to be a carcinogen (Merck, 11th ed).

Ciprofloxacin decreases the rate of ethanol elimination in humans. (1/2353)

BACKGROUND: Extrahepatic ethanol metabolism is postulated to take place via microbial oxidation in the colon, mediated by aerobic and facultative anaerobic bacteria. AIMS: To evaluate the role of microbial ethanol oxidation in the total elimination rate of ethanol in humans by reducing gut flora with ciprofloxacin. METHODS: Ethanol was administered intravenously at the beginning and end of a one week period to eight male volunteers. Between ethanol doses volunteers received 750 mg ciprofloxacin twice daily. RESULTS: A highly significant (p=0.001) reduction in the ethanol elimination rate (EER) was detected after ciprofloxacin medication. Mean (SEM) EER was 107.0 (5.3) and 96.9 (4.8) mg/kg/h before and after ciprofloxacin, respectively. Faecal Enterobacteriaceae and Enterococcus sp. were totally absent after medication, and faecal acetaldehyde production capacity was significantly (p<0.05) decreased from 0.91 (0.15) to 0.39 (0.08) nmol/min/mg protein. Mean faecal alcohol dehydrogenase (ADH) activity was significantly (p<0. 05) decreased after medication, but ciprofloxacin did not inhibit human hepatic ADH activity in vitro. CONCLUSIONS: Ciprofloxacin treatment decreased the ethanol elimination rate by 9.4%, with a concomitant decrease in intestinal aerobic and facultative anaerobic bacteria, faecal ADH activity, and acetaldehyde production. As ciprofloxacin has no effect on liver blood flow, hepatic ADH activity, or cytochrome CYP2E1 activity, these effects are probably caused by the reduction in intestinal flora.  (+info)

Successful short-term suppression of clarithromycin-resistant Mycobacterium avium complex bacteremia in AIDS. California Collaborative Treatment Group. (2/2353)

During a randomized study of clarithromycin plus clofazimine with or without ethambutol in patients with AIDS and Mycobacterium avium complex (MAC) bacteremia, eight participants received additional antimycobacterial drugs following the detection of a clarithromycin-resistant isolate (MIC, > 8 micrograms/mL). A macrolide (seven received clarithromycin, one azithromycin) and clofazimine were continued; additional treatment included various combinations of ethambutol, ciprofloxacin, amikacin, and rifabutin. After the detection of a resistant isolate and before receipt of additional antimycobacterials, the median peak MAC colony count in blood was 105 cfu/mL (range, 8-81,500 cfu/mL). After additional antimycobacterials, the median nadir MAC colony count was 5 cfu/mL (range, 0-110 cfu/mL). Five (63%) of eight patients had a > or = 1 log10 decrease, including two who achieved negative blood cultures; all of these responses occurred in patients originally assigned to clarithromycin plus clofazimine. Treatment of clarithromycin-resistant MAC bacteremia that emerges during clarithromycin-based treatment can decrease levels of bacteremia and transiently sterilize blood cultures.  (+info)

Infectious complications in 126 patients treated with high-dose chemotherapy and autologous peripheral blood stem cell transplantation. (3/2353)

The effect of an extensive prophylactic antimicrobial regimen was prospectively assessed in 126 patients after high-dose chemotherapy and autologous PBSC. They received ciprofloxacin (500 mg/12 h), acyclovir (200 mg/6 h), and itraconazole (200 mg/12 h) orally until neutrophil recovery. Febrile patients received i.v. imipenem (500 mg/6 h) to which vancomycin and amikacin were added if fever persisted for 2-3 and 5 days, respectively. Amphotericin B lipid complex was further given on day 7 or 8 of fever. Median times for a neutrophil count of >0.5 x 10(9)/l and a platelet count of >20 x 10(9)/l were 9 and 11 days. Severe neutropenia (<0.1 x 10(9)/l) lasted for a median of 5 days in which 72% of febrile episodes and 50% of cases of bacteremia occurred. Gram-positive bacteria were isolated in 30 of 40 episodes of bacteremia, 25 of which were caused by Staphylococcus epidermidis. Clinical foci were the intravascular catheter in 35 cases, respiratory infection in 11, cellulitis in two, anal abscess in one, and neutropenic enterocolitis in one. The high incidence of febrile episodes (94%) and bacteremias (31%) may be due to the lack of efficacy of antimicrobial prophylaxis and the persistence of a 5-day period of severe neutropenia.  (+info)

Prediction of the effects of inoculum size on the antimicrobial action of trovafloxacin and ciprofloxacin against Staphylococcus aureus and Escherichia coli in an in vitro dynamic model. (4/2353)

The effect of inoculum size (N0) on antimicrobial action has not been extensively studied in in vitro dynamic models. To investigate this effect and its predictability, killing and regrowth kinetics of Staphylococcus aureus and Escherichia coli exposed to monoexponentially decreasing concentrations of trovafloxacin (as a single dose) and ciprofloxacin (two doses at a 12-h interval) were compared at N0 = 10(6) and 10(9) CFU/ml (S. aureus) and at N0 = 10(6), 10(7), and 10(9) CFU/ml (E. coli). A series of pharmacokinetic profiles of trovafloxacin and ciprofloxacin with respective half-lives of 9.2 and 4 h were simulated at different ratios of area under the concentration-time curve (AUC) to MIC (in [micrograms x hours/milliliter]/[micrograms/milliliter]): 58 to 466 with trovafloxacin and 116 to 932 with ciprofloxacin for S. aureus and 58 to 233 and 116 to 466 for E. coli, respectively. Although the effect of N0 was more pronounced for E. coli than for S. aureus, only a minor increase in minimum numbers of surviving bacteria and an almost negligible delay in their regrowth were associated with an increase of the N0 for both organisms. The N0-induced reductions of the intensity of the antimicrobial effect (IE, area between control growth and the killing-regrowth curves) were also relatively small. However, the N0 effect could not be eliminated either by simple shifting of the time-kill curves obtained at higher N0s by the difference between the higher and lowest N0 or by operating with IEs determined within the N0-adopted upper limits of bacterial numbers (IE's). By using multivariate correlation and regression analyses, linear relationships between IE and log AUC/MIC and log N0 related to the respective mean values [(log AUC/MIC)average and (log N0)average] were established for both trovafloxacin and ciprofloxacin against each of the strains (r2 = 0.97 to 0.99). The antimicrobial effect may be accurately predicted at a given AUC/MIC of trovafloxacin or ciprofloxacin and at a given N0 based on the relationship IE = a + b [(log AUC/MIC)/(log AUC/MIC)average] - c [(log N0)/(log N0)average]. Moreover, the relative impacts of AUC/MIC and N0 on IE may be evaluated. Since the c/b ratios for trovafloxacin and ciprofloxacin against E. coli were much lower (0.3 to 0.4) than that for ampicillin-sulbactam as examined previously (1.9), the inoculum effect with the quinolones may be much less pronounced than with the beta-lactams. The described approach to the analysis of the inoculum effect in in vitro dynamic models might be useful in studies with other antibiotic classes.  (+info)

Influences of urinary pH on ciprofloxacin pharmacokinetics in humans and antimicrobial activity in vitro versus those of sparfloxacin. (5/2353)

The impact of acidification and alkalinization of urine on the pharmacokinetics of ciprofloxacin was investigated after single 200-mg oral doses were administered to nine healthy male volunteers. In addition, the effect of human urine on the MICs of ciprofloxacin and sparfloxacin against some common urinary tract pathogens such as Escherichia coli and Pseudomonas aeruginosa was investigated. Acidic and alkaline conditions were achieved by repeated oral doses of ammonium chloride or sodium bicarbonate, respectively. Plasma ciprofloxacin levels in all subjects were adequately described in terms of two-compartment model kinetics with first-order absorption. Acidification and alkalinization treatments had no effect on ciprofloxacin absorption, distribution, or elimination. The total amount of unchanged ciprofloxacin excreted over 24 h under acidic conditions was 88.4 +/- 14.5 mg (mean +/- standard deviation) (44.2% of the oral dose) and 82.4 +/- 16.5 mg (41.2% of the oral dose) under alkaline conditions, while the total amount of unchanged drug excreted over 24 h in volunteers receiving neither sodium bicarbonate nor ammonium chloride was 90.53 +/- 9.8 mg (45.2% of the oral dose). The mean renal clearance of ciprofloxacin was 16.78 +/- 2.67, 16.08 +/- 3.2, and 16.31 +/- 2.67 liters/h with acidification, alkalinization, and control, respectively. Renal clearance and concentrations of ciprofloxacin in urine were not correlated with urinary pH. The antibacterial activity of ciprofloxacin and sparfloxacin against E. coli NIHJ JC-2 and P. aeruginosa ATCC 27853 was affected by human urine and in particular by its pH. The activities of both quinolones against E. coli NIHJ JC-2 were lower at lower urinary pH and rather uniform, while in the case of P. aeruginosa ATCC 27853 ciprofloxacin was more active than sparfloxacin.  (+info)

Fluoroquinolone action against clinical isolates of Mycobacterium tuberculosis: effects of a C-8 methoxyl group on survival in liquid media and in human macrophages. (6/2353)

When the lethal action of a C-8 methoxyl fluoroquinolone against clinical isolates of Mycobacterium tuberculosis in liquid medium was measured, the compound was found to be three to four times more effective (as determined by measuring the 90% lethal dose) than a C-8-H control fluoroquinolone or ciprofloxacin against cells having a wild-type gyrA (gyrase) gene. Against ciprofloxacin-resistant strains, the C-8 methoxyl group enhanced lethality when alanine was replaced by valine at position 90 of the GyrA protein or when aspartic acid 94 was replaced by glycine, histidine, or tyrosine. During infection of a human macrophage model by wild-type Mycobacterium bovis BCG, the C-8 methoxyl group lowered survival 20- to 100-fold compared with the same concentration of a C-8-H fluoroquinolone. The C-8 methoxyl fluoroquinolone was also more effective than ciprofloxacin against a gyrA Asn94 mutant of M. bovis BCG. In an M. tuberculosis-macrophage system the C-8 methoxyl group improved fluoroquinolone action against both quinolone-susceptible and quinolone-resistant clinical isolates. Thus, a C-8 methoxyl group enhances the bactericidal activity of quinolones with N1-cyclopropyl substitutions; these data encourage further refinement of fluoroquinolones as antituberculosis agents.  (+info)

Comparison of efficacies of oral levofloxacin and oral ciprofloxacin in a rabbit model of a staphylococcal abscess. (7/2353)

Oral levofloxacin was compared to oral ciprofloxacin in a Staphylococcus aureus subcutaneous abscess model in rabbits. Rabbits were surgically prepared with subcutaneous wiffle balls (43 mm in diameter) and allowed to recover for 4 to 6 weeks. Rabbits were infected by direct injection into the capsule with S. aureus ATCC 29213 (5 x 10(5) CFU) and were allowed to remain infected for 8 days before the initiation of anti-infective treatment. Efficacy was determined by assessing the bacterial load within the capsule over a 10-day treatment period. In single-dose pharmacokinetic studies in infected rabbits, similar area under the concentration-time curve/MIC ratios were obtained in the plasma and abscess fluid for levofloxacin at 45 mg/kg of body weight and ciprofloxacin at 200 mg/kg of body weight. Similar efficacies were seen with levofloxacin at 45 mg/kg/day and ciprofloxacin 400 mg/kg/day by day 10. In this model, levofloxacin was significantly more efficacious than ciprofloxacin (P < 0.01).  (+info)

Pharmacodynamic comparisons of levofloxacin, ciprofloxacin, and ampicillin against Streptococcus pneumoniae in an in vitro model of infection. (8/2353)

The increasing frequency of penicillin-resistant pneumococcus continues to be of concern throughout the world. Newer fluoroquinolone antibiotics, such as levofloxacin, have shown enhanced in vitro activity against Streptococcus pneumoniae. In this study, the bactericidal characteristics and pharmacodynamic profiles of levofloxacin, ciprofloxacin, and ampicillin against four isolates of S. pneumoniae were compared by using an in vitro model of infection. Standard antibiotic dosing regimens which simulated the pharmacokinetic profile observed in humans were used. Control and treatment models were sampled for bacterial CFU per milliliter over the duration of each 24- or 48-h experiment. In addition, treatment models were sampled for MIC determinations and drug concentration. Regrowth of all isolates as well as an increase in MICs throughout the study period was observed in the ciprofloxacin experiments. A limited amount of regrowth was noted during levofloxacin therapy for one isolate; however, no change in MIC was detected for any isolate. Ampicillin showed rapid and sustained bactericidal activity against all isolates. In this study, ratios of effective fluoroquinolone area under the concentration-time curve (AUC):MIC values ranged from 30 to 55. Levofloxacin, owing to its larger AUC0-24 values, has excellent and sustained activity against different pneumococcal strains superior to that of ciprofloxacin.  (+info)

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Abstract High-pressure liquid chromatographic (HPLC) methods were developed for the analysis of ciprofloxacin hydrochloride raw materials. Method A is for drug content and the determination of related compounds eluting before the drug, including the ethylenediamine analog of ciprofloxacin. Method B...
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PAIVA, Magna Cristina et al. The first report of the qnrB19, qnrS1 and aac(6´)-Ib-cr genes in urinary isolates of ciprofloxacin-resistant Escherichia coli in Brazil. Mem. Inst. Oswaldo Cruz [online]. 2012, vol.107, n.5, pp.687-689. ISSN 0074-0276. http://dx.doi.org/10.1590/S0074-02762012000500018.. In this study, we investigated the presence of plasmid-mediated quinolone resistance (PMQR) genes among 101 ciprofloxacin-resistant urinary Escherichia coli isolates and searched for mutations in the quinolone-resistance-determining regions (QRDRs) of the DNA gyrase and topoisomerase IV genes in PMQR-carrying isolates. Eight isolates harboured the qnr and aac(6)-Ib-cr genes (3 qnrS1, 1 qnrB19 and 4 aac(6)-Ib-cr). A mutational analysis of the QRDRs in qnr and aac(6)-Ib-cr-positive isolates revealed mutations in gyrA, parC and parE that might be associated with high levels of resistance to quinolones. No mutation was detected in gyrB. Rare gyrA, parC and parE mutations were detected outside of the ...
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PCR was used to amplify a 238-bp region from Helicobacter pylori which corresponded to the quinolone resistance-determining region in Escherichia coli. The gyrA gene of H. pylori was cloned and sequenced. An open reading frame of 2,478 nucleotides coded for a polypeptide of 826 amino acids with a calculated molecular mass of 92,508 Da. The amino acid sequence showed an overall 52% identity with other bacterial gyrA genes but was most closely related to the gyrA subunit of Campylobacter jejuni (76.5% identity). Sequencing of the amplification product from ciprofloxacin-resistant mutants of H. pylori revealed four classes of mutations with substitutions at amino acid 87 (Asn--,Lys), amino acid 88 (Ala--,Val), and amino acid 91 (Asp--,Gly, --,Asn, or --,Tyr) and a double substitution at amino acids 91 and 97 (Ala--,Val). Ciprofloxacin-susceptible strains of H. pylori could be transformed to ciprofloxacin resistance by using the amplified fragment from resistant strains as donor DNA. Of the 11 ...
High-level ciprofloxacin resistance (MIC ≥ 64 μg/ml) in E. faecium is associated with amino acid changes in topoisomerase IV and DNA gyrase and confined to a single clonal complex, previously identified as a global hospital-adapted subpopulation of E. faecium (CC17). CC17 is further characterized by ampicillin resistance and a pathogenicity island, including the variant esp gene (20, 31, 32, 65) (Fig. 1). Cipr isolates belonging to other genetic backgrounds are only low-level resistant (MIC = 4 μg/ml) and do not carry these point mutations. These findings further disclose the cumulative adaptive mechanisms that have resulted in the evolution of a hospital-adapted genetic complex in E. faecium.. We have previously described the global spread of CC17 E. faecium, which is characterized by ampicillin resistance, although pandemic spread of VRE was instrumental for its identification. The strong association with quinolone resistance strongly suggests that this clonal complex has previously ...
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Objective: The objective of this study was to develop and evaluate a controlled release implant of ciprofloxacin using Bovine Hydroxyapatite-Chitosan composite and glutaraldehyde as cross-link agent.. Methods: Ciprofloxacin implants were prepared using Bovine Hydroxyapatite-Chitosan composite composition 70:30. This composite was further developed using three different concentrations of glutaraldehyde (0.5%, 0.75%, and 1,0%). Implants were formed into pellets with 4.0 mm diameters and weighed 100.0 mg using compression method. Further, the prepared ciprofloxacin implants were characterized for porosity, density, water absorption capacity, swelling ratio, degradation test, compressive strength, compatibility studies (FT-IR), morphology (SEM), X-ray diffraction study, assay, and in vitro drug release.. Results: The addition of glutaraldehyde as cross-link agent in ciprofloxacin implants showed controlled release profile of ciprofloxacin over a time period 30 d. This is caused by glutaraldehyde ...
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In our work, ciprofloxacin was extracted from the raw material ciprofloxacin hydrochloride and coordinated with the metal ions of copper, manganese and zirconium. The procedures include the comparison of the autoclave method with the solvothermal method, synthesizing the corresponding complexes and conducting antibacterial test on nearly 20 kinds of bacteria. It is shown that under the condition of PH1 and 110℃ - 116℃, the autoclave method and solvothermal method can be utilized to obtain the ciprofloxacin complexes with Cu2+, Mn2+ and Zr2+ as the metal ligands after reacting for 8 hours. The raw material, ligands and monocrystals were characterized by IR, DSC, UV and fluorescence spectrum. Meanwhile a great number of antibacterial tests were carried out, revealing favorable bacteriocidal properties of the resulting complexes.
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HPLC Application #16389: Levofloxacin and Ciprofloxacin on Luna 3µm HILIC. Column used: Luna® 3 µm HILIC 200 Å, LC Column 100 x 2 mm, Ea Part#: 00D-4449-B0
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Using data from the Gonococcal Isolate Surveillance Project, we studied changes in ciprofloxacin resistance in Neisseria gonorrhoeae isolates in the United States during 2002-2007. Compared with prevalence in heterosexual men, prevalence of ciprofloxacin-resistant N. gonorrhoeae infections showed a more pronounced increase in men who have sex with men (MSM), particularly through an increase in prevalence of strains also resistant to tetracycline and penicillin. Moreover, that multidrug resistance profile among MSM was negatively associated with recent travel. Across the surveillance project sites, first appearance of ciprofloxacin resistance in heterosexual men was positively correlated with such resistance for MSM. The increase in prevalence of ciprofloxacin resistance may have been facilitated by use of fluoroquinolones for treating gonorrhea and other conditions. The prominence of multidrug resistance suggests that using other classes of antimicrobial drugs for purposes other than treating gonorrhea
Ciprofloxacin Assay. Ciprofloxacin is an antibiotic that binds DNA topoisomerases and blocks the DNA replication process in. bacteria. The MicroGram Ciprofloxacin Assay Kit is designed for measurement of low micrograms of. ciprofloxacin. The assay is based on increase of fluorescence at 470 nm of the assay reagent in the. presence of ciprofloxacin. The assay kit can be used for measurement of ciprofloxacin concentrations in. drug discovery, drug development and pharmaceutical samples. The assay is not compatible with biological. samples containing amino acids or other molecules or buffers with amines.. ...
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Two studies13,14 in children aged 1 to 14 years with The clinical cure (91%) and bacteriologic eradication otorrhea due to P aeruginosa indicate that ciprofloxacin, (89%) rates seen in the ciprofloxacin-treated patients are 3 drops of a 0.3% solution applied topically 3 times higher than those observed after oral administration of daily for 14 days, resulted in clinical cure rates of 91% ciprofloxacin.5-7 These 3 studies, carried out in adult and 69%. Bone-conduction audiometry tests performed patients with CSOM treated with oral ciprofloxacin, 500 at baseline and at the end of treatment showed no rele- mg twice daily, showed that, at the end of the treatment, otorrhea had disappeared in 67%,5 59%,6 or 58%7 and The distribution of causal agents, similar within our two that bacterial eradication was seen in 59%, 62%, and treatment groups, showed that Staphylococcus, P aerugi- 70%, respectively. When 750 mg of oral ciprofloxacin6 nosa, and other gram-negative bacilli were the most preva- was given ...
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... is a potent inhibitor of CYP1A2, CYP2D6, and CYP3A4.[67]. Mechanism of action[edit]. Ciprofloxacin is a broad- ... Ciprofloxacin immediate-release tablets contain ciprofloxacin as the hydrochloride salt, and the XR tablets contain a mixture ... Ciprofloxacin hydrochloride (USP) is the monohydrochloride monohydrate salt of ciprofloxacin. It is a faintly yellowish to ... Hemodialysis or peritoneal dialysis can only remove less than 10% of ciprofloxacin.[56] Ciprofloxacin may be quantified in ...
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Dawson SJ, White LA (1992). "Treatment of Haemophilus aphrophilus endocarditis with ciprofloxacin". J. Infect. 24 (3): 317-20. ...
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One example is ciprofloxacin (Cipro), one of the most widely used antibiotics worldwide. Fluoroquinolones are often used for ... Bergan T; Dalhoff A; Thorsteinsson SB (1985). "A review of the pharmacokinetics and tissue penetration of ciprofloxacin": 23-36 ... Babar, S. (October 2013). "SIADH Associated With Ciprofloxacin" (PDF). The Annals of Pharmacotherapy. Sage Publishing. 47 (10 ... Frequently prescribed drugs are Avelox (moxifloxacin), Cipro (ciprofloxacin), Levaquin (levofloxacin), and, to some extent, ...
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  • The combination of ciprofloxacin and dexamethasone works by killing the bacteria that cause infection and reducing swelling in the ear. (medlineplus.gov)
  • Ciprofloxacin may also be used to reduce the amount of certain types of bacteria in the nose to prevent serious illness. (yahoo.com)
  • Ciprofloxacin fights bacteria in the body. (cigna.com)
  • Stop taking ciprofloxacin and tell your doctor right away if you have any of the following symptoms: pain/numbness/burning/tingling/weakness in your arms, hands, legs, or feet , changes in how you sense touch/pain/temperature/vibration/body position.This medication may rarely cause a severe intestinal condition ( Clostridium difficile -associated diarrhea ) due to a type of resistant bacteria. (medicinenet.com)
  • Depending on the type of bacteria present in the urinary tract and kidneys, ciprofloxacin may or may not be effective. (wisegeek.com)
  • And stopping ciprofloxacin too soon can lead to bacteria becoming resistant to it. (everydayhealth.com)
  • AURO-CIPROFLOXACIN is also used to treat inhalational anthrax (an infection caused by breathing in the spores of bacteria). (nps.org.au)
  • In the specific case of topoisomerases, some bacteria have mutated one of their amino acids so that the ciprofloxacin can only create a weak bond to the topoisomerase. (wikipedia.org)
  • Ciprofloxacin treatment can therefore potentially lead to the generation of mutations that may render bacteria resistant to ciprofloxacin. (wikipedia.org)
  • In the event of biological warfare, ciprofloxacin can be used to treat anthrax, plague, and tularemia (rabbit fever). (sourcewatch.org)
  • Ciprofloxacin injection is also used to prevent or treat plague (a serious infection that may be spread on purpose as part of a bioterror attack) and inhalation anthrax (a serious infection that may be spread by anthrax germs in the air on purpose as part of a bioterror attack). (medlineplus.gov)
  • This fact sheet provides instructions for the use of ciprofloxacin for post-exposure prophylaxis (PEP) during an emergency involving anthrax (referred to as Emergency Use Instructions (EUI) fact sheet). (cdc.gov)
  • Ciprofloxacin is FDA-approved for PEP of inhalation anthrax - to reduce the incidence or progression of disease following exposure to aerosolized Bacillus anthracis (B. anthracis) . (cdc.gov)
  • What is the usual dose of ciprofloxacin for PEP of anthrax? (cdc.gov)
  • Ciprofloxacin is also used to prevent and/or treat anthrax in people exposed to anthrax germs in the air. (yahoo.com)
  • In the event of biological warfare, ciprofloxacin may be used to treat and prevent dangerous illness that are deliberately spread such as plague, tularemia, and anthrax of the skin or mouth. (yahoo.com)
  • During an anthrax emergency, you will be given a medicine called ciprofloxacin (sip-roe-FLOX-a-sin) because you may have breathed in anthrax germs. (cdc.gov)
  • FDA is allowing certain uses of ciprofloxacin, including its use without a prescription, during an anthrax emergency. (cdc.gov)
  • Ciprofloxacin can also be used to treat people who may have been exposed to anthrax. (livestrong.com)
  • The drug, ciprofloxacin, gained popularity because it is an effective treatment for anthrax. (wisegeek.com)
  • Anyone who has possibly been exposed to anthrax can be treated with ciprofloxacin as a preventative measure. (wisegeek.com)
  • The binding of ciprofloxacin to serum proteins is 20% to 40%, which is not likely to be high enough to cause significant protein binding interactions with other drugs. (nih.gov)
  • Medical practitioners also warn of ciprofloxacin drug interactions. (wisegeek.com)
  • Concurrent administration of antacids containing magnesium hydroxide or aluminum hydroxide may reduce the bioavailability of ciprofloxacin by as much as 90% Find patient medical information for Ciprofloxacin Fluvoxamine Discount Pharmacy Oral on WebMD including its uses, side effects and safety, interactions, Windows Phone 7 Cialis Of Smartphones pictures, warnings and user ratings. (liberalconspiracy.org)
  • Proquin XR (ciprofloxacin hcl) should be administered at least 4 hours before or 2 hours after antacids containing magnesium or aluminum, sucralfate, VIDEX® (didanosine) chewable/buffered tablets or pediatric powder, metal cations such as iron, and multivitamin preparations containing zinc [see DRUG INTERACTIONS ]. (rxlist.com)
  • Concomitant administration of ciprofloxacin with milk products or calcium-fortified juices alone should be avoided since decreased absorption is possible [see DRUG INTERACTIONS ]. (rxlist.com)
  • This is a randomized, open-label, Phase 2, multi-center trial to investigate the potential interactions of ciprofloxacin and BioThrax. (clinicaltrials.gov)
  • I am easy of function, very been in discount clinical glucose and sometimes a ciprofloxacin 3 kali sehari time of infection or manifest in my cipro. (vaporizerdepot.com)
  • Ciprofloxacin injection is also used to treat patients who have fever and are at high risk for infection because they have very few white blood cells. (medlineplus.gov)
  • Ciprofloxacin - does it work for teeth infection? (drugs.com)
  • Will Ciprofloxacin cause a woman to get a yeast infection? (drugs.com)
  • Ciprofloxacin may be used with another medication to treat disseminated Mycobacterium avium complex (MAC) (a type of lung disease that often affects people with human immunodeficiency virus [HIV] ), and tuberculosis (TB) (a type of lung infection). (yahoo.com)
  • For Urinary Tract Infection I am on Ciprofloxacin for 3 days along with Pyridium for a UTI. (drugs.com)
  • Ciprofloxacin - How long does it take cipro to take affect for a kidney infection? (drugs.com)
  • Ciprofloxacin - I have finished 3 courses of Cipro for a bladder infection and it has not helped. (drugs.com)
  • KEEP taking ciprofloxacin if you have mild nausea, vomiting and/or diarrhea, a mild sunburn or a vaginal yeast infection. (cdc.gov)
  • Ciprofloxacin ear suspension is used to treat bilateral otitis media with effusion (middle ear infection with thick or sticky fluid behind the eardrum) in children undergoing surgical placement of tubes in the ears. (mayoclinic.org)
  • How Effective Is Ciprofloxacin for Kidney Infection? (wisegeek.com)
  • Ciprofloxacin for kidney infection is most effective for acute cystitis and pyelonephritis resulting from the Escherichia coli bacterium. (wisegeek.com)
  • In some cases, the risks associated with ciprofloxacin for kidney infection may outweigh its effectiveness. (wisegeek.com)
  • In such cases, ciprofloxacin for kidney infection works effectively to disarm bacterial reproduction and reduce inflammation and discomfort. (wisegeek.com)
  • I am on Ciprofloxacin for 7 days and currently on my 2nd last day of using them, it feels like the infection is slowly coming back. (webmd.com)
  • My dad was treated with ciprofloxacin when a severe upper respiratory infection turned into pneumonia last winter. (wisegeek.com)
  • One case of infection was found to be susceptible to ciprofloxacin (0.25 mg/l), piperacillin-tazobactam (1.0 mg/l), ceftriaxone (1.0 mg/l), and meropenem (0.38 mg/l). (wikipedia.org)
  • Taking ciprofloxacin increases the risk that you will develop tendinitis (swelling of a fibrous tissue that connects a bone to a muscle) or have a tendon rupture (tearing of a fibrous tissue that connects a bone to a muscle) during your treatment or for up to several months afterward. (medlineplus.gov)
  • Taking ciprofloxacin increases risk of developing tendinitis. (sourcewatch.org)
  • Both my podiatrist, who is treating my foot, and my acupuncturist, told me that ciprofloxacin is known for causing tendinitis. (peoplespharmacy.com)
  • Ciprofloxacin increases the risk of tendon ruptures and tendinitis, both during and for several months after treatment. (wisegeek.com)
  • If you experience any of the following symptoms of tendon rupture, stop taking ciprofloxacin and get emergency medical treatment: hearing or feeling a snap or pop in a tendon area, bruising after an injury to a tendon area, or inability to move or to bear weight on an affected area. (medlineplus.gov)
  • Taking ciprofloxacin may cause changes in sensation and nerve damage that may not go away even after you stop taking ciprofloxacin. (medlineplus.gov)
  • Do not stop taking ciprofloxacin, even if you feel better, until all of the medication is gone. (everydayhealth.com)
  • In the Targeted National Sewage Sludge Survey , a 2009 test of 84 samples of sewage sludge from around the U.S., the EPA found ciprofloxacin in every sample in concentrations ranging from 74.5 to 47,500 parts per billion. (sourcewatch.org)
  • Ciprofloxacin maximum serum concentrations and area under the curve are shown in the chart for the 250 mg to 1000 mg dose range. (nih.gov)
  • Ciprofloxacin serum concentrations achieved in humans served as a surrogate endpoint reasonably likely to predict clinical benefit and provided the initial basis for approval of this indication. (drugs.com)
  • Treatment of the cells with nontoxic concentrations of ciprofloxacin could maintain both stem cell morphology and clonogenicity, as well as all stem cells markers. (hindawi.com)
  • Tell your doctor if you have ever had any unusual or allergic reaction to ciprofloxacin or any other medicines. (drugs.com)
  • Ciprofloxacin may cause a severe reaction called anaphylaxis if you are allergic to it. (everydayhealth.com)
  • She presented to clinic with nausea and anorexia within a few days of addition of ciprofloxacin to her current regimen of medications, which included digoxin. (springer.com)
  • The proposed cause of the nausea and anorexia was digoxin toxicity secondary to a drug-drug interaction with ciprofloxacin. (springer.com)
  • Almost, cipro generic ciprofloxacin a acute inhibitor of the nausea is that these ounces, which should be reserved for acceptable makers that cannot once be treated, are being double overprescribed. (gastro-service-info.de)
  • The most frequent adverse reactions observed during treatment with ciprofloxacin were nausea, diarrhoea, liver function tests abnormal, vomiting, and rash. (who.int)
  • Both metronidazole and ciprofloxacin, at the valves you have listed, are compatible with obtaining, so we don't recommend pumping and depression while on these patients. (cysers.info)
  • This open study examined the efficacy and safety of combination ciprofloxacin and metronidazole for patients with active Crohn's disease of the ileum and/or colon. (hindawi.com)
  • Seventy-two patients with active Crohn's disease of the ileum (n=27), ileocolon (n=22) or colon (n=23) were treated with ciprofloxacin 500 mg bid and metronidazole 250 mg tid for a mean of 10 weeks. (hindawi.com)
  • Ciprofloxacin in combination with metronidazole is well tolerated and appears to play a beneficial role in achieving clinical remission for patients with active Crohn's disease, particularly when there is involvement of the colon. (hindawi.com)
  • Ciprofloxacin Tablets are indicated in adult patients for treatment of infectious diarrhea caused by Escherichia coli (enterotoxigenic isolates), Campylobacter jejuni, Shigella boydii † , Shigella dysenteriae, Shigella flexneri or Shigella sonnei † when antibacterial therapy is indicated. (drugs.com)
  • Ciprofloxacin Tablets are indicated for treatment of plague, including pneumonic and septicemic plague, due to Yersinia pestis (Y. pestis) and prophylaxis for plague in adults and pediatric patients from birth to 17 years of age. (drugs.com)
  • Patients receiving ciprofloxacin and digoxin should be monitored closely for the risk of digoxin toxicity. (springer.com)
  • Adequate hydration of patients receiving Proquin XR (ciprofloxacin hcl) should be maintained to prevent the formation of highly concentrated urine. (rxlist.com)
  • Patients using ciprofloxacin might also experience dizziness , nervousness, extreme fatigue, or insomnia . (wisegeek.com)
  • Risks associated with ciprofloxacin include the possibility of developing inflamed or ruptured tendons, and patients of any age may develop irritation or swelling around joints. (wisegeek.com)
  • Whenever imaginable absorption steroids, examine fingers receiving adverse time with the errand of ciprofloxacin 3 kali sehari sugar, many as ciprofloxacin review, and where full, concomitant staining. (vaporizerdepot.com)
  • Ciprofloxacin extended-reIease tablets are available in 500 mg and 1000 mg (ciprofloxacin equivalent) tablet strengths. (nih.gov)
  • Each Ciprofloxacin extended-release 500 mg tablet contains 500 mg of ciprofloxacin as ciprofloxacin HCI (287.5 mg, calculated as ciprofloxacin on the dried basis) and ciprofloxacin (212.6 mg, calculated on the dried basis). (nih.gov)
  • Ciprofloxacin given as an oral tablet is rapidly and well absorbed from the gastrointestinal tract after oral administration. (nih.gov)
  • CIPRO XR is available in 500 mg and 1000 mg (ciprofloxacin equivalent) tablet strengths. (rxlist.com)
  • Each CIPRO XR 500 mg tablet contains 500 mg of ciprofloxacin as ciprofloxacin HCl (287.5 mg, calculated as ciprofloxacin on the dried basis) and ciprofloxacin† (212.6 mg, calculated on the dried basis). (rxlist.com)
  • Each CIPRO XR 1000 mg tablet contains 1000 mg of ciprofloxacin as ciprofloxacin HCl (574.9 mg, calculated as ciprofloxacin on the dried basis) and ciprofloxacin† (425.2 mg, calculated on the dried basis). (rxlist.com)
  • Effect of chloroquine on the bioavailability of ciprofloxacin in humans," American Journal of Therapeutics , vol. 13, no. 5, pp. 432-435, 2006. (hindawi.com)
  • Gardner et al detected a breast-milk ciprofloxacin level of mg/L at about 11 hours after the last dose in a mother who had been taking mg of oral ciprofloxacin per day for 10 days. (cysers.info)
  • Oral ciprofloxacin tablets may be taken with or without food. (drugs.com)
  • This can occur after the first dose of ciprofloxacin. (medlineplus.gov)
  • You could interrupt the feeding for 1 hour before and 2 hours after the ciprofloxacin dose, or your doctor may decide to switch to a different treatment. (drugs.com)
  • A 250 mg oral dose given every 12 hours produces an AUC equivalent to that produced by an infusion of 200 mg ciprofloxacin given every 12 hours. (nih.gov)
  • Studies indicate that ciprofloxacin is only effective against these bacterial strains if they are not penicillin or methicillin resistant. (wisegeek.com)
  • Will ciprofloxacin hcl make you fail a drug test? (drugs.com)
  • consider switching either ciprofloxacin or tizanidine to an alternative drug. (cdc.gov)
  • public health officials will announce whether recipients need more ciprofloxacin and how to get additional quantities of the drug. (cdc.gov)
  • If you have had tendons get irritated or torn when taking this medicine (ciprofloxacin tablets) or an alike drug in the past. (drugs.com)
  • The Food and Drug Administration (FDA) approved ciprofloxacin in 1987 for Bayer Healthcare under the brand name Cipro. (everydayhealth.com)
  • In 2004, the FDA approved generic ciprofloxacin for several drug manufacturers. (everydayhealth.com)
  • So, the drug of choice is ciprofloxacin. (wikipedia.org)
  • Antihistamines, erythromycin, and ciprofloxacin may also cause drug-induced LQT. (wikipedia.org)
  • 4.De Schryver N, Wittebole X, Van den Bergh P, Haufroid V, Goffin E, Hantson P. Severe rhabdomyolysis associated with simvastatin and role of ciprofloxacin and amlodipine coadministration. (webmd.com)
  • Conclusion: Ciprofloxacin should be avoided initially in treating severe acute uncomplicated pyelonephritis until culture results and sensitivity is available. (scirp.org)