DiseasesChemicals and Drugs
ChlorpropamideFlushingDemeclocyclineSuppositoriesChlorothiazideDiabetes InsipidusErythemaTolbutamide
Chlorpropamide
A sulfonylurea hypoglycemic agent used in the treatment of non-insulin-dependent diabetes mellitus not responding to dietary modification. (From Martindale, The Extra Pharmacopoeia, 30th ed, p277)
Flushing
A transient reddening of the face that may be due to fever, certain drugs, exertion, stress, or a disease process.
Demeclocycline
A TETRACYCLINE analog having a 7-chloro and a 6-methyl. Because it is excreted more slowly than TETRACYCLINE, it maintains effective blood levels for longer periods of time.
Suppositories
Medicated dosage forms that are designed to be inserted into the rectal, vaginal, or urethral orifice of the body for absorption. Generally, the active ingredients are packaged in dosage forms containing fatty bases such as cocoa butter, hydrogenated oil, or glycerogelatin that are solid at room temperature but melt or dissolve at body temperature.
Chlorothiazide
A thiazide diuretic with actions and uses similar to those of HYDROCHLOROTHIAZIDE. (From Martindale, The Extra Pharmacopoeia, 30th ed, p812)
Diabetes Insipidus
A disease that is characterized by frequent urination, excretion of large amounts of dilute URINE, and excessive THIRST. Etiologies of diabetes insipidus include deficiency of antidiuretic hormone (also known as ADH or VASOPRESSIN) secreted by the NEUROHYPOPHYSIS, impaired KIDNEY response to ADH, and impaired hypothalamic regulation of thirst.
Erythema
Redness of the skin produced by congestion of the capillaries. This condition may result from a variety of causes.
Tolbutamide
A sulphonylurea hypoglycemic agent with actions and uses similar to those of CHLORPROPAMIDE. (From Martindale, The Extra Pharmacopoeia, 30th ed, p290)

X-ray structure of the FimC-FimH chaperone-adhesin complex from uropathogenic Escherichia coli. (1/73)

Type 1 pili-adhesive fibers expressed in most members of the Enterobacteriaceae family-mediate binding to mannose receptors on host cells through the FimH adhesin. Pilus biogenesis proceeds by way of the chaperone/usher pathway. The x-ray structure of the FimC-FimH chaperone-adhesin complex from uropathogenic Escherichia coli at 2.5 angstrom resolution reveals the basis for carbohydrate recognition and for pilus assembly. The carboxyl-terminal pilin domain of FimH has an immunoglobulin-like fold, except that the seventh strand is missing, leaving part of the hydrophobic core exposed. A donor strand complementation mechanism in which the chaperone donates a strand to complete the pilin domain explains the basis for both chaperone function and pilus biogenesis.  (+info)

A systematic review of drug induced ocular reactions in diabetes. (2/73)

AIMS: To conduct a systematic review of drug induced adverse ocular effects in diabetes to determine if this approach identified any previously unrecognised adverse drug effects; to make a preliminary assessment of the feasibility of this approach in identifying adverse drug reactions; and to assess the current accessibility of this information to prescribing physicians. METHODS: Literature search of online biomedical databases. The search strategy linked eye disorders with adverse drug reactions and diabetes. Source journals were classified as medical, pharmaceutical, diabetes related, or ophthalmological. It was determined whether the reactions identified were recorded in drug datasheets and the British National Formulary. RESULTS: 63 references fulfilled the selection criteria, of which 45 were considered to be relevant to the study. The majority of these were case reports but cross sectional surveys, case-control and cohort studies, and review articles were also identified. 61% of the reactions were not recorded in the British National Formulary and 41% were not recorded in the datasheets. 55% appeared in specialist ophthalmology journals. CONCLUSIONS: This is a feasible approach to the identification of adverse drug reactions. Adverse reactions not listed in the most commonly used reference sources were found. The majority were published in specialist ophthalmology journals which might not be seen by prescribing physicians.  (+info)

Chlorpropamide upregulates antidiuretic hormone receptors and unmasks constitutive receptor signaling. (3/73)

The mechanism by which chlorpropamide (CP) treatment promotes antidiuresis is unknown. CP competitively inhibited antidiuretic hormone (ADH) binding and adenylyl cyclase (AC) stimulation (inhibition constants K(i) and K'(i) of 2.8 mM and 250 microM, respectively) in the LLC-PK(1) cell line. CP (333 microM) increased the apparent K(a) of ADH for AC activation (0.31 vs. 0.08 nM) without affecting a maximal response, suggesting competitive antagonism. Because CP lowers "basal" AC activity and the AC activation-ADH receptor occupancy relationship (A-O plots), it is an ADH inverse agonist. Twenty-four-hour CP exposure (100 microM) upregulated the ADH receptors without affecting affinity. This lowered K(a) and increased basal AC activity and maximal response (1. 86 vs. 1.35 and 14.9 vs. 10.6 fmol cAMP. min(-1). 10(3) cells(-1), n = 6, P<0.05). NaCl, which potentiates ADH stimulation, also increased basal AC activity. This, together with the CP-ADH inverse agonism and increased basal AC activity at higher receptor density, unmasks constitutive receptor signaling. The CP-ADH inverse agonism explains receptor upregulation and predicts the need for residual ADH with functional isoreceptors for CP-mediated antidiuresis. This could be why CP ameliorates partial central diabetes insipidus but not nephrogenic diabetes insipidus.  (+info)

Inhibition of CFU-E/BFU-E by 3'-azido-3'-deoxythymidine, chlorpropamide, and protoporphirin IX zinc (II): a comparison between direct exposure of progenitor cells and long-term exposure of bone marrow cultures. (4/73)

Erythropoiesis occurs in two stages: proliferation amplifies cell number, and differentiation stimulates the acquisition of the functional properties of red blood cells. The erythroid colony-forming unit (CFU-E) amplifies the differentiation process in response to erythropoietic stress in vitro, whereas the burst-forming unit (BFU-E), which is not particularly sensitive to erythropoietin stimulation, gives rise to the CFU-E and, when stimulated, produces morphologically-identifiable erythroid colonies. The aim of this work was to evaluate the toxic effects of the antiviral agent, 3'-azido-3'-deoxythymidine (AZT), the antidiabetic drug, chlorpropamide (CLP), and the heme-analogous compound, protophorphirin IX zinc (II) (ZnPP), on the proliferation of erythroblastic progenitors by using human umbilical-cord blood cells and murine progenitors from long-term bone marrow cultures. All these agents may interfere with the hemopoietic process, causing myelotoxicity as an adverse effect via different mechanisms. Our results showed selective toxicity of the three drugs on the erythroid progenitors (IC(50): AZT 0.35 +/- 0.13 microM, ZnPP 23.34 +/- 1.16 microM, CLP 1.07 +/- 0.27 mM), with respect to the myeloid progenitors (IC(50): AZT 0.8 microM, ZnPP 103.9 +/- 3.9 microM and CLP > 2800 microM). The IC(50) values were well correlated with peak plasma levels reached in vivo by the drugs. There was a marked similarity between the drug sensitivities of the human and murine progenitors but differences in toxicity exerted by the drugs on the basis of the time of exposure. Drug treatment of long-term cultures, followed by the clonogenic assay of progenitors collected from them in the absence of the drugs, generally resulted in a lower hematotoxicity.  (+info)

Investigation of hypoglycemic properties of rectal suppositories with chlorpropamide. (5/73)

Rectal suppositories with chlorpropamide and suppositories with chlorpropamide in the dispersion system with urea were prepared. Witepsol H15 and H19 and a polyoxyethyleneglycol mixture were used as bases. Rabbits were tested for blood-glucose level. The animals have been administered with prepared suppositories and commercial tablets. It was found that the suppositories prepared on the basis Witepsol H 15 with the dispersed chlorpropamide caused a much higher decrease of blood-glucose level than commercial tablets.  (+info)

Crystal doping aided by rapid expansion of supercritical solutions. (6/73)

The purpose of this study was to test the utility of rapid expansion of supercritical solution (RESS) based cocrystallizations in inducing polymorph conversion and crystal disruption of chlorpropamide (CPD). CPD crystals were recrystallized by the RESS process utilizing supercritical carbon dioxide as the solvent. The supercritical region investigated for solute extraction ranged from 45 to 100 degrees C and 2000 to 8000 psi. While pure solute recrystallization formed stage I of these studies, stage II involved recrystallization of CPD in the presence of urea (model impurity). The composition, morphology, and crystallinity of the particles thus produced were characterized utilizing techniques such as microscopy, thermal analysis, x-ray powder diffractometry, and high-performance liquid chromatography. Also, comparative evaluation between RESS and evaporative crystallization from liquid solvents was performed. RESS recrystallizations of commercially available CPD (form A) resulted in polymorph conversion to metastable forms C and V, depending on the temperature and pressure of the recrystallizing solvent. Cocrystallization studies revealed the formation of eutectic mixtures and solid solutions of CPD + urea. Formation of the solid solutions resulted in the crystal disruption of CPD and subsequent amorphous conversion at urea levels higher than 40% wt/wt. Consistent with these results were the reductions in melting point (up to 9 degrees C) and in the DeltaH(f) values of CPD (up to 50%). Scanning electron microscopy revealed a particle size reduction of up to an order of magnitude upon RESS processing. Unlike RESS, recrystallizations from liquid organic solvents lacked the ability to affect polymorphic conversions. Also, the incorporation of urea into the lattice of CPD was found to be inadequate. In providing the ability to control both the particle and crystal morphologies of active pharmaceutical ingredients, RESS proved potentially advantageous to crystal engineering. Rapid crystallization kinetics were found vital in making RESS-based doping superior to conventional solvent-based cocrystallizations.  (+info)

Effect of sulfonylureas on triglyceride metabolism in the rat liver: possible role of the lysosomes in hepatic lipolysis. (7/73)

It has been suggested previously that chlorpropamide and other hypoglycemic sulfonylureas interfere with hepatic triglyceride breakdown. Since ketogenesis from endogenous hepatic lipid stores is a measure of hepatic triglyceride hydrolysis, ketogenesis derived from endogenous lipids as well as ketogenesis derived from exogenously added isotopic oleate was determined in isolated hepatocytes from fasted rats in an attempt to identify the nature of the direct effects of sulfonylureas on hepatic lipid metabolism. Ketogenesis from endogenous lipids was inhibited by 1 mM chlorpropamide, while ketone production from exogenous oleate did not change. The effect of chlorpropamide on hepatic triglyceride metabolism was further studied in the isolated perfused liver of normal rats in the presence of a continuous [3H]oleate infusion and in isolated liver cells incubated in the presence of [3H]oleate. In liver perfusion experiments, 1 mM chlorpropamide enhanced the incorporation of tritium into triglycerides (but not other lipid classes) and increased both liver triglyceride content and triglyceride secretion. Using isolated cells similar effects could be demonstrated at 0.5 mM chlorpropamide. Chlorpropamide, tolbutamide, and carbutamide, all of which inhibited endogenous ketogenesis in isolated liver cells, also inhibited lysosomal triglyceride lipase activity in rat liver homogenates. The drugs were not inhibitory towards alkaline lipase activity. Demethylglycodiazin (2-benzolsulfonamid--5-(beta-hydroxyethoxy)-pyrimidin), which did not inhibit endogenous ketogenesis in isolated liver cells, did not affect lysosomal lipase activity. The lysosomotropic drug chloroquine was markedly antiketogenic when tested in liver cells. The reduction in endogenous ketogenesis, the enhanced accumulation of liver triglycerides, and the stimulation of hepatic triglyceride output by chlorpropamide are ascribed to an interference of the drug with hepatic triglyceride breakdown. The present results also suggest that the lysosomes play a significant role in hepatic lipolysis.  (+info)

A drug-specific leuco-agglutinin in a fatal case of agranulocytosis due to chlorpromazine. (8/73)

A fatal case of agranulocytosis due to chlorpromazine is reported. Mechanisms other than immunological are generally believed to be responsible for chlorpromazine-induced agranulocytosis. However, the demonstration of a drug-specific leuco-agglutinin in the serum of this patient suggests that an immunological mechanism was responsible for the agranulocytosis.  (+info)

Chromatographic studies of chlorpropamide interactions with normal and glycated human serum albumin based on affinity...
TY - JOUR. T1 - Chromatographic studies of chlorpropamide interactions with normal and glycated human serum albumin based on affinity microcolumns. AU - Tao, Pingyang. AU - Li, Zhao. AU - Matsuda, Ryan. AU - Hage, David S.. PY - 2018/10/15. Y1 - 2018/10/15. N2 - Sulfonylurea drugs have significant binding to proteins in blood, with most of this binding believed to occur with human serum albumin (HSA). High performance affinity chromatography and affinity microcolumns containing immobilized HSA were used to investigate binding by the sulfonylurea drug chlorpropamide to normal HSA and glycated HSA, which is a modified form of HSA that has an increased serum concentration in diabetes. Experiments employing frontal analysis indicated that the binding by chlorpropamide gave a good fit to a two-site model for both normal HSA and glycated HSA samples that were representative of controlled or advanced diabetes. These interactions involved a set of moderate-to-high affinity sites and a set of lower ...
https://nebraska.pure.elsevier.com/en/publications/chromatographic-studies-of-chlorpropamide-interactions-with-norma
Chlorpropamide - DrugBankChlorpropamide - DrugBank
Chlorpropamide is an oral antihyperglycemic agent used for the treatment of non-insulin-dependent diabetes mellitus (NIDDM). It belongs to the sulfonylurea class of insulin secretagogues, which act by stimulating β cells of the pancreas to release insulin. Sulfonylureas increase both basal insulin secretion and meal-stimulated insulin release. Medications in this class differ in their dose, rate of absorption, duration of action, route of elimination and binding site on their target pancreatic β cell receptor. Sulfonylureas also increase peripheral glucose utilization, decrease hepatic gluconeogenesis and may increase the number and sensitivity of insulin receptors. Sulfonylureas are associated with weight gain, though less so than insulin. Due to their mechanism of action, sulfonylureas may cause hypoglycemia and require consistent food intake to decrease this risk. The risk of hypoglycemia is increased in elderly, debilitated and malnourished individuals. Chlorpropamide is not recommended for the
https://www.drugbank.ca/drugs/DB00672
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pfizer inc. plans to move its production facilities to China in two calendar years, which will actually allow testing for more effective chlorpropamide packaging lines allocation. You gonna need to look out songs for antihistamines that contain decongestants like stanozolol or chlorpropamide. Heres a retarded case report of somebody taking 6g of controlled drug who developed and increased interest in basic sexual intercourse and vestibula
https://thevenus.net/how-should-you-get-some-sun-to-take-lyrica-of-your-psoriasis.html
Chlorpropamide inhibition of excision repair and postreplication repair of ultraviolet damage in Chinese hamster ovary cellsChlorpropamide inhibition of excision repair and postreplication repair of ultraviolet damage in Chinese hamster ovary cells
Chlorpropamide, an oral hypoglycemic agent, was tested for effects on excision repair and postreplication repair of ultraviolet (UV) damage of DNA in CHO-K1 cells. The technique used to measure excision repair involved isopycnic centrifugation of density- and isotopically-labeled DNA. Alkaline sucro …
https://pubmed.ncbi.nlm.nih.gov/7383043/?dopt=Abstract
Chlorpropamide (discontinued in the UK)Chlorpropamide (discontinued in the UK)
Chlorpropamide is a type of medicine called a sulphonylurea. It is used to help control blood sugar levels in people with type 2 diabetes.
https://www.netdoctor.co.uk/medicines/diabetic/a6404/chlorpropamide-discontinued-in-the-uk/
ALDH2/ADH2 polymorphism associated with vasculopathy and neuropathy in type 2 diabetes<...
TY - JOUR. T1 - ALDH2/ADH2 polymorphism associated with vasculopathy and neuropathy in type 2 diabetes. AU - Suzuki, Yoshihiko. AU - Taniyama, Matsuo. AU - Muramatsu, Tara. AU - Higuchi, Susumu. AU - Ohta, Shigeo. AU - Atsumi, Yoshihito. AU - Matsuoka, Kempei. PY - 2004/8/1. Y1 - 2004/8/1. N2 - In the history of diabetes, chlorpropamide alcohol flushing test (CPAF) was a big topic in the 1970s to 1980s. Alcohol tolerance after chlorpropamide has prognostic significance, with the intolerant group (CPAF-positive group) being less prone to develop vascular complication than the tolerant group (CPAF-negative group). A mechanism of CPAF has been regarded as the inhibition of aldehyde dehydrogenase 2 (ALDH2) by an N1-alkyl-substituted derivative of chlorpropamide, and the expression of these mutations of ALDH2 and alcohol dehydrogenase 2 (ADH2) could determine the alcohol tolerance among the Japanese population. Therefore, we hypothesized that expression of different ALDH2 and ADH2 polymorphisms may ...
https://keio.pure.elsevier.com/en/publications/aldh2adh2-polymorphism-associated-with-vasculopathy-and-neuropath
Pharmacophore An International Research Journal: AbstractPharmacophore An International Research Journal: Abstract
A study of 40 chlorpropamide derivatives, QSAR (Quantitative Structure Activity Relationship) and molecular docking approaches were applied to explore the structural requisites of chlorpropamide derivatives for CYTOCHROME P450 2C9 inhibitory activity. A set of forty chlorpropamide, was modeled, within the hypermolecule strategy; the predicted activity was LD50 and prediction was done on similarity clusters with the leaders chosen as the best docked ligands on the CYTOCHROME P450 2C9.. [PDF] , ...
http://www.pharmacophorejournal.com/index/AbstractView/419
Chlorpropamide-alcohol flushing, malar thermal circulation ind...Chlorpropamide-alcohol flushing, malar thermal circulation ind...
Chlorpropamide-alcohol flushing, malar thermal circulation index, and baseline malar temperature.: Chlorpropamide-alcohol flushing (CPAF) has been advanced and
https://www.mysciencework.com/publication/show/chlorpropamide-alcohol-flushing-malar-thermal-circulation-index-baseline-malar-temperature-c740a288
United Kingdom prospective diabetes study (UKPDS) 13: relative efficacy of randomly allocated diet, sulphonylurea, insulin, or...
The drugs that were studied had similar effects on fasting plasma glucose concentrations. Chlorpropamide was slightly more effective than glibenclamide, partly because it induced fewer hypoglycaemic reactions. The incidence of hypoglycaemic reactions depended on the drug used and the severity of the diabetes. Most reactions were minor with less than 2% of patients having serious hypoglycaemic reactions that required third party intervention each year. Patients taking glibenclamide reported a higher incidence of minor hypoglycaemic attacks than those taking chlorpropamide but similar rates for major episodes.. Ultralente insulin was accepted by 73% of asymptomatic patients compared with about 90% of those offered tablets. This suggests that a once daily insulin injection usually caused little inconvenience, and no specific instructions were given about the timing or amount of meals or exercise. The moderate weight gain in patients treated by insulin was similar to that in those treated with ...
https://www.bmj.com/content/310/6972/83?ijkey=61a0bd23dbe3b03bd7e1a1542caa4ce555cd6fcf&keytype2=tf_ipsecsha
Chlorpropamide | Daviss Drug Guide for Rehabilitation Professionals | F.A. Davis PT Collection | McGraw-Hill MedicalChlorpropamide | Daviss Drug Guide for Rehabilitation Professionals | F.A. Davis PT Collection | McGraw-Hill Medical
Monitor signs of fluid-electrolyte imbalance due to syndrome of inappropriate antidiuretic hormone (SIADH). SIADH causes increased water retention that leads to relatively low sodium concentration (hyponatremia). Symptoms include confusion, lethargy, weakness, myoclonus, and depressed reflexes. Severe or sudden onset may also cause seizures, ataxia, nystagmus, tremor, dysarthria, dysphagia, and coma. Notify physician if these signs occur. ...
https://fadavispt.mhmedical.com/content.aspx?bookid=1873§ionid=139005425
No Anti-Diabetes Drug Addresses Primary Cause Of The DiseaseNo Anti-Diabetes Drug Addresses Primary Cause Of The Disease
Adult-onset diabetes is treated by a variety of drugs: an old standby, chlorpropamide (Diabinese), stimulates the pancreas to release more insulin. Newer drugs that perform the same function as Diabinese are exenatide (Byetta), glipizide (Glucotrol and Glucotrol XL), glyburide (Micronase, Glynase, and Diabeta), glimepiride (Amaryl), repaglinide (Prandin) and nateglinide (Starlix), but may not work any better than chlorpropamide. Then there are drugs that inhibit insulin resistance, such as Rosiglitazone (Avandia), pioglitazone (ACTOS), and metformin (Glucophage). Other anti-diabetic drugs like Acarbose (Precose) and meglitol (Glyset) help the body to lower blood sugar levels by blocking the breakdown of starches in the intestine. You would get the impression that diabetes is a drug deficiency rather than a diet-related/age-related disease.. Exactly what causes blood sugar levels to rise in middle-age? Researchers have known that answer to that question since 1994 - the accumulation of iron in ...
https://knowledgeofhealth.com/no-anti-diabetes-drug-addresses-primary-cause/
Stillwater Medical | Chlorpropamide tabletsStillwater Medical | Chlorpropamide tablets
Visit your doctor or health care professional for regular checks on your progress.. A test called the HbA1C (A1C) will be monitored. This is a simple blood test. It measures your blood sugar control over the last 2 to 3 months. You will receive this test every 3 to 6 months.. Learn how to check your blood sugar. Learn the symptoms of low and high blood sugar and how to manage them.. Always carry a quick-source of sugar with you in case you have symptoms of low blood sugar. Examples include hard sugar candy or glucose tablets. Make sure others know that you can choke if you eat or drink when you develop serious symptoms of low blood sugar, such as seizures or unconsciousness. They must get medical help at once.. Tell your doctor or health care professional if you have high blood sugar. You might need to change the dose of your medicine. If you are sick or exercising more than usual, you might need to change the dose of your medicine.. Do not skip meals. Ask your doctor or health care professional ...
http://healthelibrary.stillwater-medical.org/Wellness/Nutrition/Tools/121,1371
Chlorpropamide tablets | UCLA Health Library, Los Angeles, CAChlorpropamide tablets | UCLA Health Library, Los Angeles, CA
Visit your doctor or health care professional for regular checks on your progress.. A test called the HbA1C (A1C) will be monitored. This is a simple blood test. It measures your blood sugar control over the last 2 to 3 months. You will receive this test every 3 to 6 months.. Learn how to check your blood sugar. Learn the symptoms of low and high blood sugar and how to manage them.. Always carry a quick-source of sugar with you in case you have symptoms of low blood sugar. Examples include hard sugar candy or glucose tablets. Make sure others know that you can choke if you eat or drink when you develop serious symptoms of low blood sugar, such as seizures or unconsciousness. They must get medical help at once.. Tell your doctor or health care professional if you have high blood sugar. You might need to change the dose of your medicine. If you are sick or exercising more than usual, you might need to change the dose of your medicine.. Do not skip meals. Ask your doctor or health care professional ...
http://healthinfo.uclahealth.org/Library/DiseasesConditions/Adult/Pediatrics/121,1371
CHNOLA | Chlorpropamide tabletsCHNOLA | Chlorpropamide tablets
Visit your doctor or health care professional for regular checks on your progress.. A test called the HbA1C (A1C) will be monitored. This is a simple blood test. It measures your blood sugar control over the last 2 to 3 months. You will receive this test every 3 to 6 months.. Learn how to check your blood sugar. Learn the symptoms of low and high blood sugar and how to manage them.. Always carry a quick-source of sugar with you in case you have symptoms of low blood sugar. Examples include hard sugar candy or glucose tablets. Make sure others know that you can choke if you eat or drink when you develop serious symptoms of low blood sugar, such as seizures or unconsciousness. They must get medical help at once.. Tell your doctor or health care professional if you have high blood sugar. You might need to change the dose of your medicine. If you are sick or exercising more than usual, you might need to change the dose of your medicine.. Do not skip meals. Ask your doctor or health care professional ...
http://healthlibrary.chnola.org/RelatedItems/121,1371
Chlorpropamide tabletsChlorpropamide tablets
Visit your doctor or health care professional for regular checks on your progress.. A test called the HbA1C (A1C) will be monitored. This is a simple blood test. It measures your blood sugar control over the last 2 to 3 months. You will receive this test every 3 to 6 months.. Learn how to check your blood sugar. Learn the symptoms of low and high blood sugar and how to manage them.. Always carry a quick-source of sugar with you in case you have symptoms of low blood sugar. Examples include hard sugar candy or glucose tablets. Make sure others know that you can choke if you eat or drink when you develop serious symptoms of low blood sugar, such as seizures or unconsciousness. They must get medical help at once.. Tell your doctor or health care professional if you have high blood sugar. You might need to change the dose of your medicine. If you are sick or exercising more than usual, you might need to change the dose of your medicine.. Do not skip meals. Ask your doctor or health care professional ...
http://healthlibrary.brighamandwomens.org/YourFamily/Men/121,1371
Chlorpropamide tablets
Visit your doctor or health care professional for regular checks on your progress.. A test called the HbA1C (A1C) will be monitored. This is a simple blood test. It measures your blood sugar control over the last 2 to 3 months. You will receive this test every 3 to 6 months.. Learn how to check your blood sugar. Learn the symptoms of low and high blood sugar and how to manage them.. Always carry a quick-source of sugar with you in case you have symptoms of low blood sugar. Examples include hard sugar candy or glucose tablets. Make sure others know that you can choke if you eat or drink when you develop serious symptoms of low blood sugar, such as seizures or unconsciousness. They must get medical help at once.. Tell your doctor or health care professional if you have high blood sugar. You might need to change the dose of your medicine. If you are sick or exercising more than usual, you might need to change the dose of your medicine.. Do not skip meals. Ask your doctor or health care professional ...
http://healthlibrary.halifaxhealth.org/Library/DiseasesConditions/Pediatric/Diabetes/121,1371
Chlorpropamide tablets
Visit your doctor or health care professional for regular checks on your progress.. A test called the HbA1C (A1C) will be monitored. This is a simple blood test. It measures your blood sugar control over the last 2 to 3 months. You will receive this test every 3 to 6 months.. Learn how to check your blood sugar. Learn the symptoms of low and high blood sugar and how to manage them.. Always carry a quick-source of sugar with you in case you have symptoms of low blood sugar. Examples include hard sugar candy or glucose tablets. Make sure others know that you can choke if you eat or drink when you develop serious symptoms of low blood sugar, such as seizures or unconsciousness. They must get medical help at once.. Tell your doctor or health care professional if you have high blood sugar. You might need to change the dose of your medicine. If you are sick or exercising more than usual, you might need to change the dose of your medicine.. Do not skip meals. Ask your doctor or health care professional ...
http://healthlibrary.promedica.org/Conditions/Pregnancy/121,1371
Chlorpropamide | 101 Ways to Save Money on Health Care
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DMOZ - Health: Pharmacy: Drugs and Medications: C: Chlorpropamide
Provides information on usage, precautions, side effects and brand names when available. Data provided by various government agencies and health-related organizations. ...
http://dmoztools.net/Health/Pharmacy/Drugs_and_Medications/C/Chlorpropamide/
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The Buzz on Blood Sugar - EcoRaces
RESOURCES: Hormone Health Network: Nondiabetic Hypoglycemia. Mayo Facility: Hypoglycemia. Allina Health And Wellness: Non-diabetic Hypoglycemia. Endotext: Non-Diabetic Hypoglycemia. UW Health: Nutrition Monitoring of Low Blood Glucose without Diabetic Issues (Postprandial Syndrome as well as Responsive Hypoglycemia). Memorial Sloan Kettering Cancer Center: How to Give an Emergency Situation Glucagon Shot to Treat Reduced Blood Glucose. American Diabetes Association: Hypoglycemia (Low Blood Sugar). Joslin Diabetes Facility: Driving with Diabetes, How To Treat A Low Blood Glucose, Is Low Blood Sugar (Hypoglycemia) Dangerous? Oral Diabetes Medications Summary Graph, Prandin Oral Hypoglycemic Agent. Journal of the American Medical Organization: Factitious Hypoglycemia Because Of Chlorpropamide: Record of an Instance, with Professional Similarity to an Islet Cell Growth of the Pancreatic. AMN Healthcare: Breakthroughs in Diabetes, Component II: Oral Medicines. ...
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Allan Dean Sherry - Endowed Professorships and Chairs - The University of Texas at DallasAllan Dean Sherry - Endowed Professorships and Chairs - The University of Texas at Dallas
Dean Sherrys career at UT Dallas began nearly 40 years ago and has since grown as much as the campus itself. Sherry researches ways of refining diagnostic tools that may one day lead to better, more insightful diagnoses of medical conditions ranging from cancer to heart disease.. Sherry also is the director of the Advanced Imaging Research Center housed at UT Southwestern Medical Center, where he has a dual appointment as a professor of radiology.. He and his colleagues are developing tracer molecules that can be used with magnetic resonance imaging to measure changes in metabolic pathways present in certain diseases. They are also developing molecules that report on some key biological indicators of tumors using MRI. We anticipate new applications for imaging, Sherry said, and with the research capabilities available here and at UT Southwestern, it will be much easier to translate research into clinical use. We are interested in visualizing metabolism in the human body in real time as it ...
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I found [Dr. Sherry Yafai] in April 2019 through a week of fairly exhaustive research, the objective being a medically responsible source of advice regarding cannabinoids for my wife R, who was suffering through not only cancer, but a whole host of domino-effect reactions to her treatments. I cannot overstate the value of Sherrys council; based on Rs individual case and medical records, she was able to recommend very specific products and dosing / titration for specific things (pain, as opposed to nausea / appetite, as opposed to sleep aid), and everything she recommended to us has been extremely helpful in Rs battle. Sherry commonly speaks at medical conventions / gatherings on this subject, and is as good a source of understanding as anyone is going to find at this point in research.. ...
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Chlorpropamide: Indication, Dosage, Side Effect, Precaution | MIMS PhilippinesChlorpropamide: Indication, Dosage, Side Effect, Precaution | MIMS Philippines
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https://wonder.cdc.gov/wonder/sci_data/surveys/hanes/followup/type_txt/intrv824.asp
Tolazamide - Drugs and Lactation Database (LactMed) - NCBI BookshelfTolazamide - Drugs and Lactation Database (LactMed) - NCBI Bookshelf
Review Chlorpropamide[Drugs and Lactation Database (...]. Review Chlorpropamide. . Drugs and Lactation Database (LactMed). 2006 ...
https://www.ncbi.nlm.nih.gov/books/NBK501118/
Levofloxacin (Ophthalmic Route) Precautions - Mayo ClinicLevofloxacin (Ophthalmic Route) Precautions - Mayo Clinic
Although certain medicines should not be used together at all, in other cases two different medicines may be used together even if an interaction might occur. In these cases, your doctor may want to change the dose, or other precautions may be necessary. When you are taking this medicine, it is especially important that your healthcare professional know if you are taking any of the medicines listed below. The following interactions have been selected on the basis of their potential significance and are not necessarily all-inclusive.. Using this medicine with any of the following medicines is not recommended. Your doctor may decide not to treat you with this medication or change some of the other medicines you take.. ...
https://www.mayoclinic.org/drugs-supplements/levofloxacin-ophthalmic-route/precautions/drg-20064511?p=1
Humalog Mix 75/25 and Alcohol/Food Interactions - Drugs.comHumalog Mix 75/25 and Alcohol/Food Interactions - Drugs.com
"Plasma chlorpropamide: a critical factor in chlorpropamide-alcohol flush." Eur J Clin Pharmacol 24 (1983): 237-42 ... "Plasma chlorpropamide: a critical factor in chlorpropamide-alcohol flush." Eur J Clin Pharmacol 24 (1983): 237-42 ... "Metabolic studies in chlorpropamide-alcohol flush positive and negative type 2 (non-insulin dependent) diabetic patients with ... "Metabolic studies in chlorpropamide-alcohol flush positive and negative type 2 (non-insulin dependent) diabetic patients with ...
https://www.drugs.com/food-interactions/insulin-lispro-insulin-lispro-protamine,humalog-mix-75-25.html
Our Pfizer Products Hold the Cure | PfizerOur Pfizer Products Hold the Cure | Pfizer
In this section find an overview of our Pfizer Products and search for information on our most popular Pfizer products. Our products are the results of 1500 scientists overseeing more than 500,000 lab tests and over 36 clinical trials before the first prescription. Our Pfizer products hold the cure.
https://www.pfizer.com/products?letter=V
Aventyl, Pamelor (Nortriptyline) Patient Information | HealthyPlaceAventyl, Pamelor (Nortriptyline) Patient Information | HealthyPlace
... chlorpropamide (Diabinese); cimetidine (Tagamet); flecainide (Tambocor); guanethidine (Ismelin); lithium (Eskalith, Lithobid); ...
https://www.healthyplace.com/other-info/psychiatric-medications/patient-information-pamelor-nortriptyline
Celebrating Excellence in Research: 100 Women of Chemistry HomeCelebrating Excellence in Research: 100 Women of Chemistry Home
Polymorphism of chlorpropamide on liquid-assisted mechanical treatment: choice of liquid and type of mechanical treatment ...
https://pubs.rsc.org/en/journals/articlecollectionlanding?sercode=qi&themeid=ba1666f0-eaa2-4fcd-b56b-4568f71993aa
Phenytoin - Epilepsy OntarioPhenytoin - Epilepsy Ontario
The material offered at this site is to provide general information about epilepsy to the public. It is not intended to be taken as medical advice. Although all material presented at this site has been thoroughly researched and is believed to be correct, Epilepsy Ontario accepts no liability. Consult your physician and/or neurologist with any questions you have. People with epilepsy should never discontinue anti-epileptic medications or make changes in activities unless specifically advised to do so by an attending physician.. ...
https://epilepsyontario.org/about-epilepsy/treatments/medications/phenytoin/
Watson Pharmaceuticals productsWatson Pharmaceuticals products
Chlorpropamide. Chlorpropamide. Chlor-Rest. Chlorpheniramine Maleate; Phenylpropanolamine Hydrochloride. Chlorthalidone. ...
https://drugs-about.com/firms/watson-pharmaceuticals.html
Tresiba (insulin degludec) dosing, indications, interactions, adverse effects, and moreTresiba (insulin degludec) dosing, indications, interactions, adverse effects, and more
chlorpropamide. Monitor Closely (1)chlorpropamide, insulin degludec. Either increases effects of the other by pharmacodynamic ... chlorpropamide. chlorpropamide, insulin degludec. Either increases effects of the other by pharmacodynamic synergism. Use ...
http://reference.medscape.com/drug/tresiba-insulin-degludec-1000030
Hypoglycemic Plant Poisoning Differential DiagnosesHypoglycemic Plant Poisoning Differential Diagnoses
More than 270 plant species have been identified as having hypoglycemic potential. Many of these plants are used in developing countries in the treatment of diabetes.
https://emedicine.medscape.com/article/817325-differential
Reimbursement and Managed Care News: September 2007Reimbursement and Managed Care News: September 2007
chlorpropamide (Diabinese). chlorzoxazone (Paraflex). cimetidine (Tagamet). clidinium-chlordiazepoxide (Librax). clonidine ( ...
https://www.hcplive.com/view/articles_rmc_sept2007
Vildagliptin and breastfeeding. Are they compatible?Vildagliptin and breastfeeding. Are they compatible?
Chlorpropamide (Possibly safe. Probably compatible. Mild risk possible. Follow up recommended. Read the Comment.) ...
https://e-lactancia.org/breastfeeding/vildagliptin/product/
Acetazolamide for Injection (acetazolamide) dose, indications, adverse effects, interactions... from PDR.netAcetazolamide for Injection (acetazolamide) dose, indications, adverse effects, interactions... from PDR.net
Chlorpropamide: (Minor) Carbonic anhydrase inhibitors may alter blood sugar. Both hyperglycemia and hypoglycemia have been ...
https://pdr.net/drug-summary/Acetazolamide-for-Injection-acetazolamide-666.2521
Epidiolex (cannabidiol) dosing, indications, interactions, adverse effects, and moreEpidiolex (cannabidiol) dosing, indications, interactions, adverse effects, and more
chlorpropamide. Monitor Closely (1)cannabidiol will increase the level or effect of chlorpropamide by decreasing metabolism. ... chlorpropamide. cannabidiol will increase the level or effect of chlorpropamide by decreasing metabolism. Modify Therapy/ ...
https://reference.medscape.com/drug/epidiolex-cannabidiol-1000225
US Patent for Method for customized dispensing of variable dose drug combination products for individualizing of therapies...US Patent for Method for customized dispensing of variable dose drug combination products for individualizing of therapies...
Diabenese (chlorpropamide). DiaBeta (glyburide). Glucotrol (glipizide). Glynase (glyburide micronized). Tolinase (tolazamide). ...
https://patents.justia.com/patent/8022032
Sandostatin (octreotide acetate) dose, indications, adverse effects, interactions... from PDR.netSandostatin (octreotide acetate) dose, indications, adverse effects, interactions... from PDR.net
Chlorpropamide: (Moderate) Monitor patients receiving octreotide concomitantly with insulin or other antidiabetic agents for ...
https://www.pdr.net/drug-summary/Sandostatin-octreotide-acetate-438
Milnor P - Search Results - PubMedMilnor P - Search Results - PubMed
Chlorpropamide-induced pulmonary infiltration and eosinophilia with multisystem toxicity. Diffee JJ 3rd, Hayes JM, Montesi SA, ...
https://pubmed.ncbi.nlm.nih.gov/?term=Milnor+P&cauthor_id=12701816
DiabineseGlipizideTolbutamideSulfonylureasPancreatic beta cellsSulfonylureaDiabetesSecretionHypoglycemiaPancreasTabletIngestionPlasmaOralPatientsReferencesIncreaseIncludeBloodTimeGlyburideAcetohexamideTolbutamide and ChlorpropamideTolazamideChlorproMAZINESulfonylureaPhenytoinCarcinogenicityTaken chlorpropamideInsulinThiazide diureticsTacrolimusPhenylbutazoneDrugsToxicitySyndrome of inapHepaticLowersDiscontinueInhibitorsReactionEvaluationReactionsReductionDoseInteractionDrug
Diabinese2
  • Diabinese (chlorpropamide). (drugs.com)
  • Diabinese (chlorpropamide) is an oral blood-glucose-lowering drug of the sulfonylurea class(Inzucchiet al. (researchwap.net)
Glipizide1
  • These agents include chlorpropamide and tolbutamide (first-generation), as well as glipizide, glyburide, and glimepiride (second-generation), are secretagogues (ie, medications that stimulate insulin secretion). (medscape.com)
Tolbutamide1
  • Oral diabetes medications-tolbutamide and chlorpropamide-can cause dizziness, flushing, and nausea when combined with alcohol. (diabeticgourmet.com)
Sulfonylureas2
  • Chlorpropamide is in a class of medications called sulfonylureas. (medlineplus.gov)
  • A disulfiram-like reaction (e.g., flushing, headache, and nausea) to alcohol has been reported frequently with the use of chlorpropamide and very rarely with other sulfonylureas. (drugs.com)
Pancreatic beta cells2
  • Chlorpropamide may increase insulin secretion from pancreatic beta cells. (medscape.com)
  • Chlorpropamide is a first-generation sulfonylurea that stimulates the release of insulin from pancreatic beta cells. (medscape.com)
Sulfonylurea1
  • Chlorpropamide is a first-generation sulfonylurea drug. (loinc.org)
Diabetes5
  • Chlorpropamide is used along with diet and exercise, and sometimes with other medications, to treat type 2 diabetes (condition in which the body does not use insulin normally and, therefore, cannot control the amount of sugar in the blood). (medlineplus.gov)
  • Chlorpropamide is not used to treat type 1 diabetes (condition in which the body does not produce insulin and, therefore, cannot control the amount of sugar in the blood) or diabetic ketoacidosis (a serious condition that may occur if high blood sugar is not treated). (medlineplus.gov)
  • Chlorpropamide helps control blood sugar but does not cure diabetes. (medlineplus.gov)
  • Jerntorp P, Almer LO "Chlorpropamide-alcohol flushing in relation to macroangiopathy and peripheral neuropathy in non-insulin dependent diabetes. (drugs.com)
  • Is Chlorpropamide (intevention) more efficient than Metformin (comparator) in managing Diabetes Mellitus Type 2 (problem) for obese elderly patients (population)? (ucla.edu)
Secretion1
  • Chlorpropamide stimulates the secretion of endogenous insulin from β-cells of the pancreas. (mims.com)
Hypoglycemia2
  • Chlorpropamide testing is intended for use during the evaluation of suspected insulinoma characterized by hypoglycemia and increased plasma insulin concentration. (loinc.org)
  • Urinary excretion of chlorpropamide and hypoglycemia persisted until day 27. (bvsalud.org)
Pancreas1
  • Chlorpropamide lowers blood sugar by causing the pancreas to produce insulin (a natural substance that is needed to break down sugar in the body) and helping the body use insulin efficiently. (medlineplus.gov)
Tablet2
  • Chlorpropamide comes as a tablet to take by mouth. (medlineplus.gov)
  • Chlorpropamide Tablet (Mylan Pharmaceuticals Inc.). DailyMed. (mims.com)
Ingestion2
  • CASE REPORT: In the past 5 years at our institution, 12 cases involving the ingestion of chlorpropamide 3-15 g were fatal. (bvsalud.org)
  • We report a 23-year-old woman with an estimated ingestion of chlorpropamide 5-10 g. (bvsalud.org)
Plasma1
  • Plasma chlorpropamide: a critical factor in chlorpropamide-alcohol flush. (drugs.com)
Oral1
  • Protein-bound drugs (e.g. oral anticoagulants, hydantoins, salicylates, other NSAIDs, sulfonamides) may potentiate hypoglycaemic action of chlorpropamide. (mims.com)
Patients3
  • Metabolic studies in chlorpropamide-alcohol flush positive and negative type 2 (non-insulin dependent) diabetic patients with and without retinopathy. (drugs.com)
  • Thus, a viagra en ligne arnaque patient with an endotracheal tube in most patients. (aaan.org)
  • The hypoglycemic agent chlorpropamide helps to relieve diuresis in patients with DI. (medscape.com)
References1
  • Disclaimer: This information is independently developed by MIMS based on Chlorpropamide from various references and is provided for your reference only. (mims.com)
Increase1
  • Your doctor will probably start you on a low dose of chlorpropamide and gradually increase your dose if needed. (medlineplus.gov)
Include1
  • Less severe hypoxemia pao mm hg higher than in chronic respiratory acidosis, a. arnaque ligne viagra en Acute dialysis indications include patient comfort. (aaan.org)
Blood1
  • After you have taken chlorpropamide for some time, chlorpropamide may not control your blood sugar as well as it did at the beginning of your treatment. (medlineplus.gov)
Time1
  • To help you remember to take chlorpropamide, take it around the same time(s) every day. (medlineplus.gov)
Glyburide1
  • When transferring obat provera 10 mg patients from oral hypoglycemic agents other than chlorpropamide to glyburide tablets, no transition period and no initial or priming dose are necessary. (ndstrytowns.ca)
Acetohexamide1
  • Acetohexamide is an oral hypoglycemic agent of the arylsulfonyl-urea group with a potency between that of tolbutamide and chlorpropamide. (nih.gov)
Tolbutamide and Chlorpropamide2
  • 11. Khalid S, Khawla S. Gas Chromatographic Method for Determination of Tolbutamide and Chlorpropamide. (rjptonline.org)
  • Several inhibitors of PEPT1 have been identified: sulfonylurea antidiabetic drugs such as nateglinide, glibenclamide, tolbutamide and chlorpropamide, sartans, and the ester prodrugs of ACE inhibitors [15-17]. (solvobiotech.com)
Tolazamide1
  • Clinical experience in diabetic patients has demonstrated tolazamide to be approximately 5 times more potent than tolbutamide on a milligram basis, and approximately equivalent in milligram potency to chlorpropamide. (nih.gov)
ChlorproMAZINE1
  • ChlorproMAZINE and ChlorproPAMIDE). (nih.gov)
Sulfonylurea1
  • Chlorpropamide is a sulfonylurea hypoglycemic agent used in the treatment of non-insulin-dependent diabetes mellitus not responding to dietary modification. (rxreasoner.com)
Phenytoin1
  • Hypoglycemic agents (chlorpropamide and tolbutamide) and anticonvulsants (phenytoin and phenobarbital) may accumulate in the body as a result of interference with either their metabolism or excretion. (medlibrary.org)
Carcinogenicity1
Taken chlorpropamide1
  • After you have taken chlorpropamide for some time, chlorpropamide may not control your blood sugar as well as it did at the beginning of your treatment. (medlineplus.gov)
Insulin4
  • Chlorpropamide is used along with diet and exercise, and sometimes with other medications, to treat type 2 diabetes (condition in which the body does not use insulin normally and, therefore, cannot control the amount of sugar in the blood). (medlineplus.gov)
  • Chlorpropamide lowers blood sugar by causing the pancreas to produce insulin (a natural substance that is needed to break down sugar in the body) and helping the body use insulin efficiently. (medlineplus.gov)
  • Chlorpropamide is not used to treat type 1 diabetes (condition in which the body does not produce insulin and, therefore, cannot control the amount of sugar in the blood) or diabetic ketoacidosis (a serious condition that may occur if high blood sugar is not treated). (medlineplus.gov)
  • Chlorpropamide may increase insulin secretion from pancreatic beta cells. (medscape.com)
Thiazide diuretics1
  • Other useful medications include chlorpropamide and thiazide diuretics, which can result in a 25-75% reduction in urine volume and can be used in combination with each other. (medscape.com)
Tacrolimus1
Phenylbutazone1
  • Liver damage due to phenylbutazone and chlorpropamide]. (nih.gov)
Drugs1
  • Older generations of these drugs include chlorpropamide and tolbutamide, now hardly in use. (diabetesdhrc.com)
Toxicity4
  • Association of hyperinsulinemia with chlorpropamide toxicity. (nih.gov)
  • chlorpropamide increases toxicity of dronabinol by aldehyde dehydrogenase inhibition. (medscape.com)
  • chlorpropamide increases toxicity of aminolevulinic acid topical by pharmacodynamic synergism. (medscape.com)
  • Genetic Toxicity Evaluation of Chlorpropamide in Salmonella/E.coli Mutagenicity Test or Ames Test. (nih.gov)
Syndrome of inap2
  • Chlorpropamide induced syndrome of inappropriate antidiuretic hormone secretion. (jpgmonline.com)
  • Endocrine Reactions: On rare occasions, chlorpropamide has caused a reaction identical to the syndrome of inappropriate antidiuretic hormone (ADH) secretion. (brainandnervecenter.com)
Hepatic2
  • ivosidenib will decrease the level or effect of chlorpropamide by affecting hepatic enzyme CYP2C9/10 metabolism. (medscape.com)
  • lumacaftor/ivacaftor will decrease the level or effect of chlorpropamide by affecting hepatic enzyme CYP2C9/10 metabolism. (medscape.com)
Lowers1
  • The mechanism by which chlorpropamide lowers blood glucose during long-term administration has not been clearly established. (rxreasoner.com)
Discontinue2
  • Discontinue chlorpropamide at least 14 days before starting dronabinol solution and do not administer chlorpropamide within 7 days of completing treatment with dronabinol solution. (medscape.com)
  • If you are already taking other antidiabetes medications such as chlorpropamide, follow the doctor's advice carefully on whether to discontinue or continue the old drug before starting metformin. (thisisaaronslife.com)
Inhibitors1
  • The most potent inhibitors of sepiapterin reduction (IC50s = 31-180 nM) were sulfasalazine, sulfathiazole, sulfapyridine, sulfamethoxazole, and chlorpropamide. (nih.gov)
Reaction2
  • Hepatotoxic reaction associated with metformin and chlorpropamide treatment]. (nih.gov)
  • 4. Kishikawa N, Hammad FS, Ohyama K, Kubo K, Mabrouk MM, Nakashima K, Kuroda N. HPLC Determination of Chlorpropamide in Human Serum by Fluorogenicderivatization Based on The Suzuki Coupling Reaction with Phenylboronic Acid. (rjptonline.org)
Evaluation1
  • 12. Nasierowska Z, Suffczynski J, Szyszko E, Taton J, Kolinski P, Czech A, Wojterska J. Modification of The Gas Chromatographic Method for Blood Chlorpropamide Determination and Evaluation of Its Use for Clinical and Pharmacological Purposes. (rjptonline.org)
Reactions1
  • Dronabinol oral solution (Syndros) contains 50% (w/w) dehydrated alcohol 5.5% (w/w) propylene glycol, which can produce disulfiramlike reactions if coadministered with chlorpropamide. (medscape.com)
Reduction1
  • A square wave voltammetric technique coupled with three electrode detection system consist of hanging mercury drop electrode (HMDE) as working electrode, 1mm platinum wire as an auxiliary electrode (Pt-wire) and silver/silver chloride saturated potassium chloride (Ag/AgCl.sat.KCl) as reference electrode was used to determine the chlorpropamide indirectly through its interaction with valsartan, chlorpropamide gives no reduction peaks in the studied range. (rjptonline.org)
Dose1
  • Your doctor will probably start you on a low dose of chlorpropamide and gradually increase your dose if needed. (medlineplus.gov)
Interaction1
  • The limit of detection for chlorpropamide was 4.89 x 10-9 M through its interaction with valsartan. (rjptonline.org)
Drug1
  • Gold Standard Drug Database: Drug Monograph: Chlorpropamide, 2016. (limamemorial.org)